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-- phpMyAdmin SQL Dump
-- version 4.0.10.18
-- https://www.phpmyadmin.net
--
-- Host: localhost:3306
-- Generation Time: Feb 20, 2017 at 08:24 AM
-- Server version: 5.5.54-cll
-- PHP Version: 5.6.30

SET SQL_MODE = "NO_AUTO_VALUE_ON_ZERO";
SET time_zone = "+00:00";


/*!40101 SET @OLD_CHARACTER_SET_CLIENT=@@CHARACTER_SET_CLIENT */;
/*!40101 SET @OLD_CHARACTER_SET_RESULTS=@@CHARACTER_SET_RESULTS */;
/*!40101 SET @OLD_COLLATION_CONNECTION=@@COLLATION_CONNECTION */;
/*!40101 SET NAMES utf8 */;

--
-- Database: `jsurgaca_medicine`
--

-- --------------------------------------------------------

--
-- Table structure for table `access`
--

CREATE TABLE IF NOT EXISTS `access` (
  `aid` int(11) NOT NULL AUTO_INCREMENT,
  `mask` varchar(255) NOT NULL DEFAULT '',
  `type` varchar(255) NOT NULL DEFAULT '',
  `status` tinyint(4) NOT NULL DEFAULT '0',
  PRIMARY KEY (`aid`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=2 ;

--
-- Dumping data for table `access`
--

INSERT INTO `access` (`aid`, `mask`, `type`, `status`) VALUES
(1, '%@%.peacled.xyz', 'mail', 0);

-- --------------------------------------------------------

--
-- Table structure for table `accesslog`
--

CREATE TABLE IF NOT EXISTS `accesslog` (
  `aid` int(11) NOT NULL AUTO_INCREMENT,
  `sid` varchar(64) NOT NULL DEFAULT '',
  `title` varchar(255) DEFAULT NULL,
  `path` varchar(255) DEFAULT NULL,
  `url` text,
  `hostname` varchar(128) DEFAULT NULL,
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  `timer` int(10) unsigned NOT NULL DEFAULT '0',
  `timestamp` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`aid`),
  KEY `accesslog_timestamp` (`timestamp`),
  KEY `uid` (`uid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `actions`
--

CREATE TABLE IF NOT EXISTS `actions` (
  `aid` varchar(255) NOT NULL DEFAULT '0',
  `type` varchar(32) NOT NULL DEFAULT '',
  `callback` varchar(255) NOT NULL DEFAULT '',
  `parameters` longtext NOT NULL,
  `description` varchar(255) NOT NULL DEFAULT '0',
  PRIMARY KEY (`aid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `actions`
--

INSERT INTO `actions` (`aid`, `type`, `callback`, `parameters`, `description`) VALUES
('backup_migrate_action_backup', '', 'backup_migrate_action_backup', '', ''),
('casetracker_actions_take_case_action', 'node', 'casetracker_actions_take_case_action', '', 'Take over case'),
('comment_publish_action', 'comment', 'comment_publish_action', '', 'Publish comment'),
('comment_unpublish_action', 'comment', 'comment_unpublish_action', '', 'Unpublish comment'),
('node_make_sticky_action', 'node', 'node_make_sticky_action', '', 'Make post sticky'),
('node_make_unsticky_action', 'node', 'node_make_unsticky_action', '', 'Make post unsticky'),
('node_promote_action', 'node', 'node_promote_action', '', 'Promote post to front page'),
('node_publish_action', 'node', 'node_publish_action', '', 'Publish post'),
('node_save_action', 'node', 'node_save_action', '', 'Save post'),
('node_unpromote_action', 'node', 'node_unpromote_action', '', 'Remove post from front page'),
('node_unpublish_action', 'node', 'node_unpublish_action', '', 'Unpublish post'),
('user_block_ip_action', 'user', 'user_block_ip_action', '', 'Ban IP address of current user'),
('user_block_user_action', 'user', 'user_block_user_action', '', 'Block current user'),
('workflow_select_next_state_action', 'node', 'workflow_select_next_state_action', '', 'Change workflow state of post to next state');

-- --------------------------------------------------------

--
-- Table structure for table `actions_aid`
--

CREATE TABLE IF NOT EXISTS `actions_aid` (
  `aid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  PRIMARY KEY (`aid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `advanced_help_index`
--

CREATE TABLE IF NOT EXISTS `advanced_help_index` (
  `sid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `module` varchar(255) NOT NULL DEFAULT '',
  `topic` varchar(255) NOT NULL DEFAULT '',
  `language` varchar(12) NOT NULL DEFAULT '',
  PRIMARY KEY (`sid`),
  KEY `language` (`language`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=222 ;

--
-- Dumping data for table `advanced_help_index`
--

INSERT INTO `advanced_help_index` (`sid`, `module`, `topic`, `language`) VALUES
(1, 'strongarm', 'README.txt', 'en'),
(2, 'autoload', 'README.txt', 'en'),
(3, 'rdf', 'README.txt', 'en'),
(4, 'advanced_help', 'using-advanced-help', 'en'),
(5, 'advanced_help', 'translation', 'en'),
(6, 'advanced_help', 'ini-file', 'en'),
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(15, 'content', 'rearrange', 'en'),
(16, 'content', 'remove', 'en'),
(17, 'content', 'theme', 'en'),
(18, 'content', 'theme-node-templates', 'en'),
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(26, 'ckeditor', 'README.txt', 'en'),
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(57, 'date_api', 'date-argument', 'en'),
(58, 'date_api', 'date-filter', 'en'),
(59, 'date_api', 'embedding', 'en'),
(60, 'date_api', 'date-repeat-form', 'en'),
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(62, 'date_api', 'date-api-functions', 'en'),
(63, 'date_api', 'form-elements', 'en'),
(64, 'date_api', 'date-timezones', 'en'),
(65, 'date_api', 'date-views-dates', 'en'),
(66, 'diff', 'readme.txt', 'en'),
(67, 'document_cloud', 'README.txt', 'en'),
(68, 'emaudio', 'README.txt', 'en'),
(69, 'emimage', 'README.txt', 'en'),
(70, 'emvideo', 'README.txt', 'en'),
(71, 'emfield', 'README.txt', 'en'),
(72, 'feeds', 'README.txt', 'en'),
(73, 'filefield', 'README.txt', 'en'),
(74, 'flag', 'README.txt', 'en'),
(75, 'flag_actions', 'README.txt', 'en'),
(76, 'flickrapi', 'README.txt', 'en'),
(77, 'gmap', 'setting up', 'en'),
(78, 'gmap', 'filter', 'en'),
(79, 'gmap', 'api', 'en'),
(80, 'gmap', 'array', 'en'),
(81, 'gmap', 'extending', 'en'),
(82, 'ie_css_optimizer', 'README.txt', 'en'),
(83, 'imageapi', 'README.txt', 'en'),
(84, 'imageapi_gd', 'README.txt', 'en'),
(85, 'imagecache', 'README.txt', 'en'),
(86, 'imagecache_ui', 'README.txt', 'en'),
(87, 'imagefield', 'README.txt', 'en'),
(88, 'imce', 'README.txt', 'en'),
(89, 'imce_mkdir', 'README.txt', 'en'),
(90, 'install_profile_api', 'README.txt', 'en'),
(91, 'jcarousel', 'README.txt', 'en'),
(92, 'job_scheduler', 'README.txt', 'en'),
(93, 'jquery_ui', 'README.txt', 'en'),
(94, 'l10n_update', 'README.txt', 'en'),
(95, 'libraries', 'README.txt', 'en'),
(96, 'login_destination', 'README.txt', 'en'),
(97, 'mimemail', 'README.txt', 'en'),
(98, 'modalframe', 'README.txt', 'en'),
(99, 'morelikethis_taxonomy', 'README.txt', 'en'),
(100, 'nodeblock', 'README.txt', 'en'),
(101, 'nodequeue', 'about', 'en'),
(102, 'nodequeue', 'smartqueue', 'en'),
(103, 'nodequeue', 'api', 'en'),
(104, 'calais_api', 'README.txt', 'en'),
(105, 'paging', 'README.txt', 'en'),
(106, 'panels', 'api', 'en'),
(107, 'panels', 'plugins-layout', 'en'),
(108, 'panels', 'plugins-style', 'en'),
(109, 'premium', 'README.txt', 'en'),
(110, 'scheduler', 'README.txt', 'en'),
(111, 'tabs', 'README.txt', 'en'),
(112, 'token_profile', 'README.txt', 'en'),
(113, 'unique_field', 'README.txt', 'en'),
(114, 'userprotect', 'README.txt', 'en'),
(115, 'viewscarousel', 'README.txt', 'en'),
(116, 'views_slideshow_singleframe', 'page-slideshow', 'en'),
(117, 'views_slideshow_singleframe', 'image-slideshow', 'en'),
(118, 'views_slideshow', 'README.txt', 'en'),
(119, 'views_slideshow_ddblock', 'README.txt', 'en'),
(120, 'workflow', 'README.txt', 'en'),
(121, 'admin', 'README.txt', 'en'),
(122, 'imagecrop', 'README.txt', 'en'),
(123, 'noderelationships', 'README.txt', 'en'),
(124, 'pathauto', 'README.txt', 'en'),
(125, 'auto_nodetitle', 'README.txt', 'en'),
(126, 'conference', 'readme.txt', 'en'),
(127, 'nodewords', 'README.txt', 'en'),
(128, 'calais', 'README.txt', 'en'),
(129, 'token', 'README.txt', 'en'),
(130, 'views', 'about', 'en'),
(131, 'views', 'getting-started', 'en'),
(132, 'views', 'example-users-by-role', 'en'),
(133, 'views', 'example-recent-stories', 'en'),
(134, 'views', 'example-user-feed', 'en'),
(135, 'views', 'example-author-block', 'en'),
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(137, 'views', 'display', 'en'),
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(139, 'views', 'display-page', 'en'),
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(147, 'views', 'relationship', 'en'),
(148, 'views', 'style', 'en'),
(149, 'views', 'style-row', 'en'),
(150, 'views', 'style-list', 'en'),
(151, 'views', 'style-unformatted', 'en'),
(152, 'views', 'style-table', 'en'),
(153, 'views', 'style-grid', 'en'),
(154, 'views', 'style-rss', 'en'),
(155, 'views', 'style-fields', 'en'),
(156, 'views', 'style-node', 'en'),
(157, 'views', 'style-node-rss', 'en'),
(158, 'views', 'style-comment-rss', 'en'),
(159, 'views', 'style-summary-unformatted', 'en'),
(160, 'views', 'style-summary', 'en'),
(161, 'views', 'menu', 'en'),
(162, 'views', 'path', 'en'),
(163, 'views', 'analyze-theme', 'en'),
(164, 'views', 'using-theme', 'en'),
(165, 'views', 'theme-css', 'en'),
(166, 'views', 'overrides', 'en'),
(167, 'views', 'embed', 'en'),
(168, 'views', 'new', 'en'),
(169, 'views', 'updating', 'en'),
(170, 'views', 'api', 'en'),
(171, 'views', 'api-tables', 'en'),
(172, 'views', 'api-default-views', 'en'),
(173, 'views', 'api-handlers', 'en'),
(174, 'views', 'api-plugins', 'en'),
(175, 'views', 'api-example', 'en'),
(176, 'morelikethis', 'README.txt', 'en'),
(177, 'calais_geo', 'README.txt', 'en'),
(178, 'features', 'README.txt', 'en'),
(179, 'devel', 'README.txt', 'en'),
(180, 'devel', 'README_devel_node_access.txt', 'en'),
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(186, 'page_manager', 'getting-started-custom-nodes', 'en'),
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(190, 'page_manager', 'custom-pages-access', 'en'),
(191, 'page_manager', 'custom-pages-menu', 'en'),
(192, 'page_manager', 'variants', 'en'),
(193, 'jquery_update', 'README.txt', 'en'),
(195, 'content_profile', 'README.txt', 'en'),
(196, 'autoassignrole', 'README.txt', 'en'),
(197, 'backup_migrate', 'README.txt', 'en'),
(198, 'casetracker', 'README.txt', 'en'),
(199, 'casetracker_basic', 'README.txt', 'en'),
(200, 'dashboard', 'README.txt', 'en'),
(201, 'lightbox2', 'README.txt', 'en'),
(202, 'uploadpath', 'README.txt', 'en'),
(203, 'role_delegation', 'README.txt', 'en'),
(204, 'accordion_menu', 'README.txt', 'en'),
(205, 'apply_for_role', 'README.txt', 'en'),
(206, 'node_import', 'introduction', 'en'),
(207, 'node_import', 'new-import', 'en'),
(208, 'node_import', 'input-format', 'en'),
(209, 'node_import', 'supported', 'en'),
(210, 'node_import', 'supported-core', 'en'),
(211, 'node_import', 'supported-cck', 'en'),
(212, 'node_import', 'supported-contrib', 'en'),
(213, 'node_import', 'examples', 'en'),
(214, 'token_array', 'README.txt', 'en'),
(215, 'views_cloud', 'README.txt', 'en'),
(216, 'views_or', 'README.txt', 'en'),
(217, 'node_form_rearrange', 'README.txt', 'en'),
(218, 'admin_theme', 'README.txt', 'en'),
(219, 'email', 'README.txt', 'en'),
(220, 'rules_forms', 'README.txt', 'en'),
(221, 'views_data_export', 'README.txt', 'en');

-- --------------------------------------------------------

--
-- Table structure for table `authmap`
--

CREATE TABLE IF NOT EXISTS `authmap` (
  `aid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `uid` int(11) NOT NULL DEFAULT '0',
  `authname` varchar(128) NOT NULL DEFAULT '',
  `module` varchar(128) NOT NULL DEFAULT '',
  PRIMARY KEY (`aid`),
  UNIQUE KEY `authname` (`authname`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `autoassignrole`
--

CREATE TABLE IF NOT EXISTS `autoassignrole` (
  `arid` varchar(38) NOT NULL DEFAULT '',
  `value` text,
  PRIMARY KEY (`arid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `autoassignrole`
--

INSERT INTO `autoassignrole` (`arid`, `value`) VALUES
('auto_active', '1'),
('auto_admin_active', '0'),
('auto_roles', 'a:5:{i:5;i:5;i:3;i:0;i:4;i:0;i:8;i:0;i:7;i:0;}'),
('node_user_register', ''),
('page_active', '0'),
('user_active', '0'),
('user_description', ''),
('user_fieldset_title', 'Manuscript submission'),
('user_multiple', '0'),
('user_required', '0'),
('user_roles', 'a:5:{i:5;i:5;i:3;i:0;i:4;i:0;i:8;i:0;i:7;i:0;}'),
('user_selection', '2'),
('user_sort', 'SORT_ASC'),
('user_title', 'Roles');

-- --------------------------------------------------------

--
-- Table structure for table `autoassignrole_page`
--

CREATE TABLE IF NOT EXISTS `autoassignrole_page` (
  `rid` int(11) NOT NULL,
  `display` text NOT NULL,
  `path` varchar(255) NOT NULL,
  `menu` varchar(255) NOT NULL,
  `title` text NOT NULL,
  `description` longtext,
  `format` tinyint(4) DEFAULT NULL,
  `weight` tinyint(4) DEFAULT '0',
  `registration` tinyint(4) DEFAULT '0',
  PRIMARY KEY (`rid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `autoassignrole_page`
--

INSERT INTO `autoassignrole_page` (`rid`, `display`, `path`, `menu`, `title`, `description`, `format`, `weight`, `registration`) VALUES
(5, '2', 'user/register/author', 'primary-links', 'Registration', '', 1, 0, 1);

-- --------------------------------------------------------

--
-- Table structure for table `backup_migrate_destinations`
--

CREATE TABLE IF NOT EXISTS `backup_migrate_destinations` (
  `destination_id` varchar(32) NOT NULL DEFAULT '0',
  `name` varchar(255) NOT NULL,
  `type` varchar(32) NOT NULL,
  `location` text NOT NULL,
  `settings` text NOT NULL,
  PRIMARY KEY (`destination_id`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `backup_migrate_profiles`
--

CREATE TABLE IF NOT EXISTS `backup_migrate_profiles` (
  `profile_id` varchar(32) NOT NULL DEFAULT '0',
  `name` varchar(255) NOT NULL,
  `filename` varchar(50) NOT NULL,
  `append_timestamp` tinyint(3) unsigned NOT NULL DEFAULT '0',
  `timestamp_format` varchar(14) NOT NULL,
  `filters` text NOT NULL,
  PRIMARY KEY (`profile_id`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `backup_migrate_schedules`
--

CREATE TABLE IF NOT EXISTS `backup_migrate_schedules` (
  `schedule_id` varchar(32) NOT NULL DEFAULT '0',
  `name` varchar(255) NOT NULL,
  `source_id` varchar(32) NOT NULL DEFAULT 'db',
  `destination_id` varchar(32) NOT NULL DEFAULT '0',
  `profile_id` varchar(32) NOT NULL DEFAULT '0',
  `keep` int(11) NOT NULL DEFAULT '0',
  `period` int(11) NOT NULL DEFAULT '0',
  `last_run` int(11) NOT NULL DEFAULT '0',
  `enabled` tinyint(3) unsigned NOT NULL DEFAULT '0',
  `cron` tinyint(3) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`schedule_id`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `batch`
--

CREATE TABLE IF NOT EXISTS `batch` (
  `bid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `token` varchar(64) NOT NULL,
  `timestamp` int(11) NOT NULL,
  `batch` longtext,
  PRIMARY KEY (`bid`),
  KEY `token` (`token`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `blocks`
--

CREATE TABLE IF NOT EXISTS `blocks` (
  `bid` int(11) NOT NULL AUTO_INCREMENT,
  `module` varchar(64) NOT NULL DEFAULT '',
  `delta` varchar(32) NOT NULL DEFAULT '0',
  `theme` varchar(64) NOT NULL DEFAULT '',
  `status` tinyint(4) NOT NULL DEFAULT '0',
  `weight` tinyint(4) NOT NULL DEFAULT '0',
  `region` varchar(64) NOT NULL DEFAULT '',
  `custom` tinyint(4) NOT NULL DEFAULT '0',
  `throttle` tinyint(4) NOT NULL DEFAULT '0',
  `visibility` tinyint(4) NOT NULL DEFAULT '0',
  `pages` text NOT NULL,
  `title` varchar(64) NOT NULL DEFAULT '',
  `cache` tinyint(4) NOT NULL DEFAULT '1',
  PRIMARY KEY (`bid`),
  UNIQUE KEY `tmd` (`theme`,`module`,`delta`),
  KEY `list` (`theme`,`status`,`region`,`weight`,`module`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=455 ;

--
-- Dumping data for table `blocks`
--

INSERT INTO `blocks` (`bid`, `module`, `delta`, `theme`, `status`, `weight`, `region`, `custom`, `throttle`, `visibility`, `pages`, `title`, `cache`) VALUES
(1, 'user', '0', 'garland', 1, 0, 'left', 0, 0, 0, '', '<none>', -1),
(2, 'user', '1', 'garland', 1, 0, 'left', 0, 0, 0, '', '', -1),
(3, 'system', '0', 'garland', 1, 10, 'footer', 0, 0, 0, '', '', -1),
(4, 'system', '0', 'openpublish_theme', 0, 38, '', 0, 0, 0, '', '', -1),
(5, 'user', '0', 'openpublish_theme', 1, 28, 'logoarea', 0, 0, 0, '', '<none>', -1),
(6, 'user', '1', 'openpublish_theme', 0, 16, '', 0, 0, 0, '', '', -1),
(7, 'block', '1', 'openpublish_theme', 0, -25, '', 0, 0, 0, '', '', -1),
(8, 'block', '4', 'openpublish_theme', 0, -17, '', 0, 0, 1, '', '', -1),
(9, 'block', '5', 'openpublish_theme', 0, -16, '', 0, 0, 0, '', '', -1),
(10, 'block', '3', 'openpublish_theme', 0, 24, '', 0, 0, 0, '', '', -1),
(11, 'block', '2', 'openpublish_theme', 0, 29, '', 0, 0, 0, '', '', -1),
(12, 'blog', '0', 'openpublish_theme', 0, 22, '', 0, 0, 0, '', '', 1),
(13, 'comment', '0', 'openpublish_theme', 0, 23, '', 0, 0, 0, '', '', 1),
(14, 'locale', '0', 'openpublish_theme', 0, -8, '', 0, 0, 0, '', '', -1),
(15, 'menu', 'admin', 'openpublish_theme', 0, -33, '', 0, 0, 0, '', '', -1),
(16, 'menu', 'devel', 'openpublish_theme', 0, -23, '', 0, 0, 0, '', '', -1),
(17, 'menu', 'features', 'openpublish_theme', 0, -20, '', 0, 0, 0, '', '', -1),
(18, 'menu', 'menu-footer-primary', 'openpublish_theme', 0, -19, '', 0, 0, 0, '', '', -1),
(19, 'menu', 'menu-footer-secondary', 'openpublish_theme', 0, -18, '', 0, 0, 0, '', '', -1),
(20, 'menu', 'primary-links', 'openpublish_theme', 0, 20, '', 0, 0, 0, '', '', -1),
(21, 'menu', 'secondary-links', 'openpublish_theme', 0, 25, '', 0, 0, 0, '', '', -1),
(22, 'menu', 'menu-top-menu', 'openpublish_theme', 0, 30, '', 0, 0, 0, '', '', -1),
(23, 'node', '0', 'openpublish_theme', 0, 27, '', 0, 0, 0, '', '', -1),
(24, 'profile', '0', 'openpublish_theme', 0, -30, '', 0, 0, 1, 'admin/conference/paper', 'My Profile', 5),
(25, 'search', '0', 'openpublish_theme', 0, -40, '', 0, 0, 0, '', 'SEARCH', -1),
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-- --------------------------------------------------------

--
-- Table structure for table `blocks_roles`
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--
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('views', 'author-block_4', 7),
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-- --------------------------------------------------------

--
-- Table structure for table `boxes`
--

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--
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(2, '<embed src="/sites/all/files/images/banner3.swf" width="728" height="90"></embed>', 'Top Banner Ad', 2),
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(7, '<center><script type="text/javascript" src="http://widget.supercounters.com/flag.js"></script><script type="text/javascript">sc_flag(231937,"F3F3F3","000000","cccccc",2,20,0,0)</script><br><noscript><a href="http://www.supercounters.com/">Flag Counter</a></noscript>\r\n</center>\r\n\r\n', 'supercounter', 2),
(8, 'Medical Research & Innovation Secretariat\r\nUniversiti Kebangsaan Malaysia Medical Centre\r\nLevel 15, Pre-Clinical Block, Jalan Yaacob Latif\r\nBandar Tun Razak, 56000 Cheras\r\nKuala Lumpur, Malaysia.\r\n\r\n<b>Phone:</b>\r\nTel: 03-91458638/ 03-91459499\r\nFax: 03-91456634\r\n  \r\n<b>Email:</b>\r\nmedhealthukm@gmail.com', 'contact', 2),
(9, '<div><script src="http://www.widgeo.net/geocompteur/geocompteur.php?c=geoipod_w&id=2031832&adult=0&cat=sante&fonce=&claire="></script><br>\r\n<noscript><a href="http://www.widgeo.net">widgets</a></noscript>\r\n</div>', 'counter', 2),
(10, '<a href=user/register class=register title=''Join M&H to receive updates''></a>\r\n', 'regsiter', 2),
(11, '<a href=/user/register/author class=aregister title=''Join M&H as an author''></a>', 'Author Registration', 1),
(12, '<h1>ABOUT MEDICINE & HEALTH</h1>\r\n<p style="text-align:justify;"><i>Medicine and Health</i> is an international journal published by the Faculty of Medicine Universiti Kebangsaan Malaysia. The journal is published twice in a year in electronic format. The journal publishes invited editorials, reviews, original research papers, interesting case reports and clinical quiz. There are no publication charges. All submitted manuscripts are subjected to rigorous peer review process done by at least two independent reviewers in the specialized field. Articles are published in English language. In addition, an abstract in Bahasa Malaysia is also published for all the articles except editorials and clinical quiz. All articles are freely accessible in electronic format.</p>', 'front about', 2),
(13, '<a href=/node/add/conference-paper class=aregister title=''Submit manuscript online''></a>', 'Submit paper', 1),
(14, '<p style=text-align:center;><img src="/sites/medicineandhealthukm.com/files/images/indexed_in.png" ><br>\r\n<img src="/sites/medicineandhealthukm.com/files/images/esci.png">&nbsp;&nbsp;<a href=http://www.drji.org/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logo_index_drji.png"></a><br><a href=http://www.doaj.org/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logo_index_doaj.jpg"></a>&nbsp;&nbsp;<a href=http://www.cas.org/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logo_index_cas.jpg"><a href=>&nbsp;&nbsp;<a href=http://www.wprim.org/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logo_index_wprim.jpg"></p>\r\n<p align=center><a href=http://sindexs.org/JournalList.aspx?ID=1296/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/SIS_logo.png"></a><a href=http://www.base-search.net/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logo_base.gif"></a></p>\r\n<p><a href=http://crossref.org/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/crossref.jpg"></a>&nbsp;&nbsp;<a href=https://www.ebscohost.com/title-lists/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/ebscohost.png"></a></p><p><a href=http://www.mycite.my/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/images.png"></a>&nbsp;&nbsp;<a href=http://healthsciences.academickeys.com/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/academic-keys.png"></a>&nbsp;&nbsp;<a href=http://www.researchbib.com/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logoRB.png"></a></p>\r\n<p><a href=http://jgateplus.com/search/login/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/logojgate.png"></a>&nbsp;&nbsp;<a href=http://scholar.google.com/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/googlelogo2.png"></a><a href=http://clarivate.com/ target=blank><img src="/sites/medicineandhealthukm.com/files/images/index clarivate 2.jpg"></a></p>\r\n\r\n', 'indexby', 2),
(15, '<p  style="background:#eee;padding:15px;">Register with M&H to :\r\n- view full article\r\n- download pdf file\r\n- store/bookmark article\r\n- receive updates</p>\r\n\r\n<a href=user/register class=register title=''Join M&H to receive updates''></a>', 'register to view', 2),
(16, '<a href=/node/add/conference-paper><img src=/sites/all/files/images/submit_paper.png title="Submit manuscript" /></a>', 'submit online', 2);

-- --------------------------------------------------------

--
-- Table structure for table `calais_geo`
--

CREATE TABLE IF NOT EXISTS `calais_geo` (
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `center_tid` int(10) unsigned DEFAULT NULL,
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  `height` varchar(255) DEFAULT NULL,
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) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `calais_geo_term`
--

CREATE TABLE IF NOT EXISTS `calais_geo_term` (
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) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `calais_node_queue`
--

CREATE TABLE IF NOT EXISTS `calais_node_queue` (
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) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `calais_term`
--

CREATE TABLE IF NOT EXISTS `calais_term` (
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  KEY `tdid` (`tdid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `calais_term_data_company`
--

CREATE TABLE IF NOT EXISTS `calais_term_data_company` (
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  PRIMARY KEY (`did`),
  UNIQUE KEY `tid` (`tid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `calais_term_data_geo`
--

CREATE TABLE IF NOT EXISTS `calais_term_data_geo` (
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  PRIMARY KEY (`did`),
  UNIQUE KEY `tid` (`tid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `calais_term_data_product`
--

CREATE TABLE IF NOT EXISTS `calais_term_data_product` (
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  UNIQUE KEY `tid` (`tid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `calais_term_node`
--

CREATE TABLE IF NOT EXISTS `calais_term_node` (
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) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `casetracker_case`
--

CREATE TABLE IF NOT EXISTS `casetracker_case` (
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  `pid` int(11) NOT NULL DEFAULT '0',
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  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`),
  KEY `p_id` (`pid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `casetracker_case`
--

INSERT INTO `casetracker_case` (`nid`, `vid`, `pid`, `case_number`, `assign_to`, `case_priority_id`, `case_type_id`, `case_status_id`) VALUES
(30, 33, 29, 0, 3, 2, 11, 4);

-- --------------------------------------------------------

--
-- Table structure for table `casetracker_case_states`
--

CREATE TABLE IF NOT EXISTS `casetracker_case_states` (
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  `weight` tinyint(4) NOT NULL DEFAULT '0',
  PRIMARY KEY (`csid`),
  KEY `weight` (`weight`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=12 ;

--
-- Dumping data for table `casetracker_case_states`
--

INSERT INTO `casetracker_case_states` (`csid`, `case_state_name`, `case_state_realm`, `weight`) VALUES
(1, 'High', 'priority', -1),
(2, 'Normal', 'priority', 0),
(3, 'Low', 'priority', 1),
(4, 'Open', 'status', 0),
(5, 'Resolved', 'status', 1),
(6, 'Deferred', 'status', 2),
(7, 'Duplicate', 'status', 3),
(8, 'Closed', 'status', 4),
(9, 'Revision', 'type', 0),
(10, 'Feature Request', 'type', 1),
(11, 'General Task', 'type', 2);

-- --------------------------------------------------------

--
-- Table structure for table `casetracker_comment_status`
--

CREATE TABLE IF NOT EXISTS `casetracker_comment_status` (
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  `pid` int(11) NOT NULL DEFAULT '0',
  `title` varchar(250) DEFAULT NULL,
  `assign_to` int(11) NOT NULL DEFAULT '0',
  `case_priority_id` int(11) NOT NULL DEFAULT '0',
  `case_type_id` int(11) NOT NULL DEFAULT '0',
  `case_status_id` int(11) NOT NULL DEFAULT '0',
  `state` int(11) NOT NULL DEFAULT '0',
  KEY `cid` (`cid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `ckeditor_role`
--

CREATE TABLE IF NOT EXISTS `ckeditor_role` (
  `name` varchar(128) NOT NULL DEFAULT '',
  `rid` int(11) NOT NULL DEFAULT '0',
  PRIMARY KEY (`name`,`rid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `ckeditor_role`
--

INSERT INTO `ckeditor_role` (`name`, `rid`) VALUES
('Advanced', 3),
('Advanced', 4),
('Advanced', 8),
('Default', 3);

-- --------------------------------------------------------

--
-- Table structure for table `ckeditor_settings`
--

CREATE TABLE IF NOT EXISTS `ckeditor_settings` (
  `name` varchar(128) NOT NULL DEFAULT '',
  `settings` text,
  PRIMARY KEY (`name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `ckeditor_settings`
--

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INSERT INTO `conference` (`pnid`, `pvid`, `ruid`, `rnid`, `comment1`, `comment2`, `status`) VALUES
(761, 766, 1, 896, '<p>\r\n	testing</p>\r\n', '<p>\r\n	author</p>\r\n', 0),
(761, 764, 1, 896, '<p>\r\n	testing</p>\r\n', '<p>\r\n	author</p>\r\n', 0),
(900, 905, 4, 978, 'to the editor', 'to the author', 0),
(900, 905, 86, 948, 'test', 'test', 0),
(761, 766, 4, 965, 'test 2', 'test 2', 0),
(903, 908, 89, 904, '<p>\r\n	testing abc 12345 abc</p>\r\n', '<p>\r\n	testing 123</p>\r\n', 0),
(903, 908, 1, 0, '', '', 0),
(951, 956, 98, 0, '', '', 0),
(951, 956, 97, 952, 'An interesting case report. A few grammatical errors that need correction. Interesting figures as well but need to be labelled with arrows to point out the features described. Figure 1 is however of poor quality and do not depict the features described well. My suggestions and comments are included as track changes in the manuscript.', 'Interesting case report with good figures attached. However, there are a few grammatical errors and the figures should be labelled with arrows to point out the features described e.g. where the worm is. Figure 1 is however not satisfactory as it does not highlight clearly the features described.', 0),
(907, 912, 4, 908, 'TO THE EDITOR', 'TO THE AUTHOR', 0),
(946, 951, 86, 947, 'kfjkljmhkmhh,l;', 'rjiogjpokopjhiojokopk', 0),
(949, 954, 94, 0, '', '', 0),
(761, 766, 86, 0, '', '', 0),
(959, 964, 97, 960, 'Good and interesting study. A few comments which I have included in the manuscript as track changes.\r\n\r\nAbstract: Not kept to 250 words.\r\n\r\nResults: One of the results may be wrong. Page 13, the score for photostability for combination with vitamin C and E is higher than either alone. Therefore it should be higher photostability.\r\n\r\nFigures: Can be improved. Line graph in figure 1 wrongly labelled.\r\n\r\nDiscussion: Ok\r\n\r\nConclusion: Last line confusing. Author needs to elaborate.\r\n', 'Good and interesting study. A few comments which I have included in the manuscript as track changes.\r\n\r\nAbstract: Not kept to 250 words.\r\n\r\nResults: One of the results may be wrong. Page 13, the score for photostability for combination with vitamin C and E is higher than either alone. Therefore it should be higher photostability.\r\n\r\nFigures: Can be improved. Line graph in figure 1 wrongly labelled.\r\n\r\nDiscussion: Ok\r\n\r\nConclusion: Last line confusing. Author needs to elaborate.\r\n', 0),
(959, 964, 108, 962, 'This manuscript on the use of sunscreens has enough scientific merit to justify its publication in Medicine and health. However, there are, as always, numerous grammatical and spelling errors. I have only highlighted a FRACTION of the errors, which I have edited myself, as there are just too many to edit. I suggest that the authors subject their manuscript to professional editing to improve the standard of English used.', 'This is an interesting manuscript describing the results of an experiment comparing local and imported sunscreens, with and without the antioxidants vitamins C and E. The manuscript needs some editing in terms of its language. Some, but not all of the errors in the manuscript have been corrected, and these have been annotated in the manuscript itself. For instance, I am not sure that the word "Adjuvantion" in the title exists in the English dictionary. I also felt that the authors should enter some background information on the previous use of vitamins C and E in sunscreens at the end of the introduction, to show the rationale of doing the experiment, and that they are not simply re-inventing the wheel. The most obvious error, of course, is the absence of page numbers.', 0),
(956, 961, 109, 964, 'Reviewer’s Comments (27 March 2013)\r\nImpact on Knowledge in Dengue Management among Medical and Paramedical personnel following attendance to Educational Lectures (MS 0010)\r\nGeneral:\r\nThe introduction ought to have highlighted the series of educational programmes as proposed by the Ministry of Health; it appears as though the educational lecture that was given is just a “one-off” attempt at providing CME to health care personnel and the main drawback of this paper is as though it was part of an “audit” /feedback of lecture content at the end of any ‘ordinary concept lecture’.  There isn’t much new information that is derived based on this study – giving a “didactic’ concept lecture as part of a CME is indeed widely practised but its effectiveness is always a concern. As such since the authors themselves raised the issue of having an interactive session, what new ideas or approaches can be incorporated to make the CME more effective? This would have added depth to the study. The results of doctors having better basic knowledge than paramedics is “reassuring” but this probably isn’t surprising anyway – so conclusions derived at the end of this study probably does not add on to any “new” information.\r\nPERSONAL COMMENT TO EDITORIAL BOARD:\r\n\r\nWhilst I understand that the journal requires papers for publication, the quality of research papers that are finally accepted should not be compromised. As such, my overall impression of this research is that of an ‘audit/feedback” at the end of any ordinary CME that has been conducted. The study is superficially conducted and no new elements are actually highlighted. \r\n', 'Reviewer’s Comments (27 March 2013)\r\nImpact on Knowledge in Dengue Management among Medical and Paramedical personnel following attendance to Educational Lectures (MS 0010)\r\nGeneral:\r\nThe introduction ought to have highlighted the series of educational programmes as proposed by the Ministry of Health; it appears as though the educational lecture that was given is just a “one-off” attempt at providing CME to health care personnel and the main drawback of this paper is as though it was part of an “audit” /feedback of lecture content at the end of any ‘ordinary concept lecture’.  There isn’t much new information that is derived based on this study – giving a “didactic’ concept lecture as part of a CME is indeed widely practised but its effectiveness is always a concern. As such since the authors themselves raised the issue of having an interactive session, what new ideas or approaches can be incorporated to make the CME more effective? This would have added depth to the study. The results of doctors having better basic knowledge than paramedics is “reassuring” but this probably isn’t surprising anyway – so conclusions derived at the end of this study probably does not add on to any “new” information.\r\nDetails:\r\n1.	The introduction and review of literature was too generic. \r\n2.	In the methods section, there was no mention of when the study was conducted and who were the actual sample population – ie was this carried out at a district/health or general hospital level as there will be variability in the respondents who attended. Also was it the same person who delivered the talk – this is important as the level of experience and knowledge of the speaker will influence the results of this study\r\n3.	How was the content of questionnaire actually derived? Target group discussion or...?  Mere extraction from the CPG is probably not the best way to go, esp if no additional discussions was carried out between relevant individuals. This needs to be highlighted. The pretest was done on both doctors and supporting staff or...please clarify. Presumably it was delivered in English only or....? If so, will that have any restrictions (comprehension) on the paramedic personnel.....are nurses included in this group or...? Not entirely clear.\r\n4.	Why was a cut off point of 1 week taken to analyse post K? \r\n5.	How were these same 61 respondents captured again at post K level...?.please clarify. \r\n6.	Concept of questionnaire validity is not correct. \r\n7.	Results were not entirely clear – details of the questionnaire and scoring system used ought to have been highlighted further. \r\n8.	When discussing results, it is the important facts or gaps in knowledge that ought to be highlighted rather than what was reported as high percentages (page 4 –last paragraph). \r\n9.	Conclusion/recommendation – as mentioned, does not add on to any “new” information; maybe better to suggest if other CME modalities can be incorporated eg e-update/learning package on line as the study suggests that on-going CME helps with consolidation of core knowledge; so its these “innovative” suggestions that will add on ‘weight” to this manuscript\r\n10.	References – not alphabetically arranged and name of journal ought to be italicised\r\n11.	Style of writing really needs to be addressed. Suggest for a proof-reader to read and check the spelling and especially the grammar first prior to submission to any journal. Quite a significant amount of correction has been mentioned in the manuscript itself.\r\n12.	 **Additional notes are also mentioned briefly in the Reviewers’ Checklist that has been submitted.\r\n\r\n\r\n', 0),
(956, 961, 110, 0, '', '', 0),
(971, 976, 115, 0, '', '', 0),
(971, 976, 116, 0, '', '', 0),
(975, 980, 108, 0, '', '', 0),
(975, 980, 133, 0, '', '', 0),
(976, 981, 113, 0, '', '', 0),
(977, 982, 113, 0, '', '', 0),
(972, 977, 135, 0, '', '', 0),
(972, 977, 136, 0, '', '', 0),
(968, 973, 98, 0, '', '', 0),
(968, 973, 91, 0, '', '', 0),
(961, 966, 108, 0, '', '', 0),
(974, 979, 115, 0, '', '', 0),
(974, 979, 142, 0, '', '', 0),
(974, 979, 143, 0, '', '', 0),
(976, 981, 148, 0, '', '', 0),
(977, 982, 148, 0, '', '', 0),
(979, 984, 86, 980, 'testing', 'testing', 0),
(974, 979, 150, 981, 'The paper basically lacks cohesiveness from the introduction to discussion. The data collected is valid but has to be studied from the correct perspective. It is unwise to define healthy life-style as just the alcohol and smoking. Healthy life-style must and should include (at least) diet, exercise. Hence the focus of the paper should just be about smoking and alcohol.', 'The paper has to be re-written with a more appropriate perspective: a focus on alcohol and smoking. Combining both and defining the two health behaviours as unhealthy behaviour misled the readers as healthy behaviour is more than just the two, and should at minimum, to include diet and exercise. furthermore, combining them in the analysis with just a model will dilute out the determinants'' effect. ', 0),
(979, 984, 81, 0, '', '', 0),
(761, 766, 81, 0, '', '', 0),
(900, 905, 81, 0, '', '', 0),
(972, 977, 97, 982, 'This is practically just an audit which does not have much merit. That being said, it should still be of interest to the paramedics and those on duty at sport events such as silat. Language editing needed as there are a number of grammatical errors. Table 1 and 3 total injuries are not the same. List of essential items should be written as Table 4. There is no reference to the tables in the text. There is also no list of references at the end of the manuscript. References quoted are too few ( only 3). The format of referencing in the text does not follow the journal''''s requirements. More references and discussion needed if it is to be published as an original article. Otherwise it would be appropriate as only a letter to the editor.\r\n\r\n', 'Language editing needed as there are a number of grammatical errors. Table 1 and 3 total injuries are not the same. List of essential items should be written as Table 4. There is no reference to the tables in the text. There is also no list of references at the end of the manuscript. References quoted are too few ( only 3). The format of referencing in the text does not follow the journal''''s requirements. More references and discussion needed if it is to be published as an original article.', 0),
(972, 977, 151, 983, 'Dear Editor,\r\nMy apology for the delay in sending my review of this manuscript. I have to conclude that this manuscript does not meet the standard of our Journal. It is actually an audit of what transpired during an event. It did not meet the criteria of a retrospective review as mentioned. It also did meet its own objective of bringing improvement to the way the sport is being handled. There was not a single reference quoted too. As such I recommend to reject this manuscript.\r\nkind regards,\r\nAP Dr. Dayang A Aziz', 'Dear Authors,\r\nI find your manuscript as an audit of your performance at this event. Common types of blunt injuries are expected in any contact sports. Your manuscript did not give any added value to what we already know. Based on your report, it was evident that the way the medical team has managed this event was already appropriate and your "study" did not provide any change in the protocol. I strongly feel that an audit such as this is not suitable for publication in this journal. It did not meet the requirement for this journal.\r\nkind regards,\r\nReviewer.', 0),
(903, 908, 81, 0, '', '', 0),
(977, 982, 163, 0, '', '', 0),
(961, 966, 151, 0, '', '', 0),
(961, 966, 133, 0, '', '', 0),
(987, 993, 150, 7238, 'good paper, but I have suggested some critical analysis and clearer method description', 'recommend revision. see file attached.', 0),
(987, 993, 108, 1021, 'Please ensure that there is a neurologist among the authors. This is because all the patients were cared for by neurologists during the acute phase of stroke in the wards; the neurologists are an integral part of the management of stroke and the conclusions drawn from this study will reflect the standard of care rendered by them. The standard of English used is also mediocre and needs major editing. However, I am happy for it to be published if the suggested corrections are made.', 'This manuscript presents the results of a study of an increasingly important aspect of stroke management. However, the sample size is small and the strength of the conclusions from this study is affected by this limitation. There are also numerous errors throughout the manuscript, including errors in spelling, grammar and the use of capital letters for non-proper nouns, such as the word "Ischemic". The entire manuscript has NO PAGE NUMBERS and spaces are frequently missing between words. There are also statements made in the manuscript which are not based on the study itself but from conjecture. These have been indicated by annotations made directly onto the manuscript. There are also missing words and unknown terminologies. ', 0),
(1070, 1077, 108, 1071, 'This is a report of a questionnaire-based survey; although there is no strong analytical component to this research, I think it is worth publishing it for others to read. The level of English is poor.', 'An informative piece of work that is worth disseminating to other healthcare workers. The paper needs substantial editing in terms of grammar, syntax and spelling. There should be consistency in the use of the terminology "respondents", and not interchange it with "participants". I am not sure what "Primer Health Clinic" is; it is probably best to use "Primary Health Clinic". Sentences should not start with digits; if they do, the digits must be spelt out in full. Paroxysmal atrial fibrillation is also a risk factor for stroke and so a regular pulse on examination does not exclude paroxysmal atrial fibrillation and this risk factor for stroke.', 0),
(1067, 1074, 224, 0, '', '', 0),
(1154, 1162, 207, 0, '', '', 0),
(1213, 1221, 91, 0, '', '', 0),
(1213, 1221, 207, 0, '', '', 0),
(1282, 1290, 207, 1286, 'Dear editor,\r\nThe manuscript may be revised and submitted again.\r\nRegards,\r\nSD', 'The manuscript has been well written.\r\nPlease begin the abstract with a background sentence.\r\nPlease add limitations of study.\r\nHow many observers took part? How was error minimized?\r\nPlease add few latest references from 2013/2014/2015.', 0),
(1271, 1279, 207, 0, '', '', 0),
(1285, 1293, 207, 0, '', '', 0),
(1293, 1301, 233, 0, '', '', 0),
(1291, 1299, 268, 0, '', '', 0),
(1300, 1308, 207, 0, '', '', 0),
(6547, 6556, 97, 7233, 'Quite difficult to understand. A number of spelling and grammatical errors. Even the abstract in Malay has mistakes. Need to be corrected properly by their supervisor/lecturer before it can be reviewed again. At this current stage, would be embarrassing to publish this paper.', 'An interesting paper but there are a lot of spelling and grammatical errors. Please get your supervisor and lecturer or someone who has a good grasp of English to review and correct the paper and then resubmit.', 0),
(7244, 7253, 1749, 0, '', '', 0),
(7310, 7319, 113, 0, '', '', 0),
(7330, 7339, 1884, 25493, 'Acceptable but requires grammar and format corrections', 'the manuscript requires grammar corrections and reformatting \r\n', 0),
(13141, 13150, 1885, 25504, 'This article can be considered provided corrections are made based of the track changes and recommendations.', 'Good review but probably needs extra assistance on the English grammar.  It would be beneficial to readers to understand the 3 conditions ie os trigonum, pes cavus,  pes planus with a diagram.   \r\n\r\nTable 1 is not mentioned in text\r\n', 0),
(7319, 7328, 211, 0, '', '', 0),
(13184, 13193, 211, 0, '', '', 0),
(13184, 13193, 97, 25639, 'Excellent work. However, there are a few comments:\r\n1. Figure 1 MMP3 no significant difference between active and inactive disease (p=0.268). No explanation given.\r\n2. Abstract in Malay obviously google translated with many errors. Cannot understand at all. Please rewrite.\r\n3. In methods, the name of the MMP3 detection kit needs to be described in detail.\r\n\r\n', 'Excellent work. However, there are a few comments:\r\n1. Figure 1 MMP3 no significant difference between active and inactive disease (p=0.268). No explanation given.\r\n2. Abstract in Malay obviously google translated with many errors. Cannot understand at all. Please rewrite.\r\n3. In methods, the name of the MMP3 detection kit needs to be described in detail.', 0),
(25501, 25510, 2063, 0, '', '', 0),
(25501, 25510, 135, 0, '', '', 0),
(7354, 7363, 1884, 29403, 'Acceptable but requires grammar corrections.\r\n', 'Title & Abstract: \r\n‘Expression of p53 protein in premenopausal age women as risk factors for breast cancer ‘.  ‘age’  can be removed to shorten the title. Capitalise each word.\r\n\r\nAbstrak: …. oleh (Immunohistochemistry)IHC...’IHC’ should be the one in bracket.\r\n.... kategori proteein....correct the spelling.\r\n\r\nAbstract: Please standardise the way the value is presented. Add 0 before the decimal eg 0.08, 0.52…. Standardise to 2 decimal place only.\r\n\r\nIntroduction:\r\n\r\nLots of grammatical errors that requires checking. \r\nPlease use past tense.\r\nPlease use the abbreviation once it has been defined eg IHC. Do not repeat the full term.\r\n\r\nAmong them:\r\n\r\nIn normal cell cycles (add a comma) of proliferation and regression are controlled by….\r\n\r\nA Malaysian women’s cumulative risk of breast cancer in her lifetime is in the ratio of (1:20) with the highest risk of  occurring in Chinese women (1:14)\r\n\r\n\r\nMaterial & Methods/Case:\r\n\r\nPlease use past tense to describe the methodology.\r\n\r\nLast paragraph – incomplete sentence: ‘See Figure 1 for’\r\n\r\n\r\n\r\nResults:  \r\n\r\nStyle of presenting data could be improved. Limit to 2 decimal place. Add 0 before decimal.\r\n\r\nConflicting statement:\r\n‘In addition, majority of the respondents aged 41 years and less also have positive p53 (+) protein category (n= 3/111)’. Should be minority n=3\r\nDiscussion\r\n\r\nGood discussion but need to check the grammar.\r\n\r\n\r\nConclusion\r\n\r\nToo long. Limitations of the study in the second paragraph should be moved to Discussion section.\r\n\r\nFigures and Tables\r\nStandardise the values: Limit to 2 decimal place. Add 0 before decimal.\r\n\r\nThe photomicrograph is in black and white. Cannot appreciate the be brownish precipitations.\r\nWhy is the magnification not standardized. X10 or X40\r\nWhat is S0045464 and other similar labels? Please mention them in the footnote of the Table.\r\n\r\n\r\n\r\nCritical comments:\r\nthe manuscript requires grammar corrections by an expert in English', 0),
(27848, 27858, 207, 0, '', '', 0),
(25681, 25691, 207, 0, '', '', 0),
(27866, 27876, 1754, 0, '', '', 0),
(29682, 29692, 207, 0, '', '', 0),
(29692, 29702, 97, 29796, 'Acceptable', 'Well written', 0),
(29574, 29584, 1884, 29847, 'Acceptable. Need to correct grammatical errors.', 'A good study which provides information on CAM uses among diabetic patients. Two main findings were the use of bitter gourd and that patients do not reveal their CAM use to their doctors.\r\nThe manuscript requires grammar checking.', 0),
(29574, 29584, 207, 0, '', '', 0),
(28918, 28928, 207, 0, '', '', 0),
(29687, 29697, 207, 0, '', '', 0),
(29954, 29964, 163, 0, '', '', 0),
(32689, 32699, 207, 0, '', '', 0),
(29959, 29969, 1884, 41164, 'This is a good case report due to the extremely rare nature of the disease. However , the manuscript needs to be checked for its grammar by a person with good English.', 'Reporting a rare disease. Need to check back the grammar.', 0),
(53295, 53307, 207, 0, '', '', 0),
(42869, 42879, 233, 0, '', '', 0),
(46084, 46094, 2428, 56584, 'Dear Editor,\r\n\r\nI would recommend to reject this article based on the following points:\r\n\r\n-	The manuscript suffers major errors in view of the inappropriate title, study design and the results obtained.\r\n-	The manuscript does not in line with the scope of Medicine & Heath.\r\n-	The results (MIC and MFC values) was found to be the same in a previously published article:  Khosravi AR, Shokri H, Mokhtari AR. Efficacy of Cuminum cyminum essential oil on FUM1 gene expression of fumonisin-producing Fusarium verticillioides strains. Avicenna J Phytomed. 2015;5(1):34-42.\r\n\r\nRegards,\r\nTeoh SL', 'Title & Abstract:\r\nThe title is not suitable: there is no results showing the function of FUM1 gene in this manuscript. \r\n\r\nScientific names should be italicized: Fusarium verticillioides.\r\nThe abstract should be in a single paragraph, and no abbreviations should be used.\r\nThis section should be rewritten to summarise the manuscript better, especially in the methodology and result part.\r\nThe authors have not mentioned the treatment of Cuminum cyminum essential oil to the isolates.\r\nIn addition, a conclusion is required for the abstract.\r\n\r\nCorrections:\r\n-	Fusarium is one of the most important pathogens > Fusarium verticillioides is one…\r\n-	soil nitrogen contentincrease > increased in soil nitrogen content\r\n-	3 Fusarium non verticillioides samples (negative control) > 3 non Fusarium verticillioides samples\r\n-	For testing MIC and MFC, the RPMI1640 culture in microplate was used > The antifungal  effect of Cuminum cyminum essential oil was determined by broth microdilution testing.\r\n-	the cultured RNA samples using the RNA extraction kit of Fermentascompany (Purification Mini Kit GeneJET Plant RNA) was extracted from fungal biomass > RNA was extracted from the fungal biomass\r\n-	RT-PCR test along with the FUM1 gene was performed > RT-PCR test for the detection of FUM1 gene expression was performed\r\n-	FUM1 gene was expressed in fungal samples that were not under the effects of oil essence > FUM1 gene was expressed in all fungal samples\r\n-	gene was not expressed in samples after the cumin impact and there was not any band in agarose gel > gene was not expressed following Cuminum cyminum essential oil treatment\r\n\r\nIntroduction:\r\nThis section needs major re-arrangement of text.\r\n\r\nMajor issues:\r\nThe authors should focus on the background to the research and its aims: i.e. Fusarium verticillioides, Cuminum cyminum essential oil and fumonisin. Infact, the authors have burden the introduction section with categorization of fungus, environmental conditions promoting fungal growth, history of micotoxicology and the methodology of analyzing gene expression. \r\n\r\nCorrections:\r\n-	consumption of contaminated food of fungal > consumption of fungal-contaminated food\r\n-	so that the consumer countries have put food safety in top priority and the technical aspects of this issue have overcome its economic aspects > the authors need to rephrase this sentence.\r\n-	micotoxin pollution categories have been added to the indicators of the quality of the food > micotoxin pollution have been included as an indicator of food quality.\r\n-	Fungi is categorized in the new classification apart from plants and animals > Fungi is categorized in a different kingdom from plants and animals\r\n-	To classify fungal food spoilage their appearance would also considered > the authors need to rephrase this sentence.\r\n-	 The fungus colonized different parts of the plant and it often asymptomatic lesions. > The fungus colonized in different parts of the plant and often present as asymptomatic lesions.\r\n-	due to weather conditions, physiological conditions and the amount of nitrogen > due to changes in weather and physiological conditions, and increased amount of nitrogen in soil.  \r\n-	Important characteristics such as pigment > Important characteristic features of  Fusarium species are presence of pigment\r\n-	25 °-C > 25°C\r\n-	Although a variety of Fusarium are capable of producing fumonisin, but most of them are unknown, and they differ in terms of toxin production. > the authors needs to clarify this statement, and are required to explain further on what is fumonisin. \r\n-	In the explanation of Cumin (Cuminum cyminum), the authors have explained the characteristic of its roots and leaves twice: i.e. ‘its root is lanky and white’ and ‘Its root is spindly, more or less branched with 10-20 cm long, lanky and white’; ‘its leaves are thin and stringy with green color’ and ‘Its leaves are alternate, transparent, hairless, leading to very thin incisions and delicate, but it is long and stringy. The leaves are dark green color and its base attached to the stem by a more or less flat sheath’.\r\n\r\nMaterial & Methods/Case:\r\nAuthors should describe the experimental methods in enough detail that other researchers can replicate results and evaluate claims.\r\n\r\nMajor issues:\r\n-	In this part, the authors did not include the methodology on how the Cuminum cyminum essential oil is produced (or maybe purchased), how the treatment of Cuminum cyminum essential oil was performed in the experiment, fungi RNA isolation, and the RT-PCR protocol.\r\n-	The authors have included the methodology of DNA extraction, what is the rationale of performing DNA extraction when the authors have extracted RNA and performed RT-PCR to obtain cDNA?\r\n-	The microscopic analysis of fungi was also not mentioned in this section although figures were presented in the result section.\r\n\r\nCorrections:\r\n-	the plates were incubated for 48 h at 35 ° > 35°C\r\n-	scores of 2 (50 percent), scores of 3 (75 percent) > 50%, 75%\r\n-	The PCR experiments was conducted on extracted RNA to assess DNA contamination> experiments were \r\n-	The PCR experiments was conducted on extracted RNA to assess DNA contamination > the authors have mentioned the use of DNAse to remove contaminated DNA, and not by PCR.\r\n-	DNase with amount of lμ5 > 5μl\r\n-	The test is performed in a volume of 25-ml > It is impossible for PCR to have a 25 ml reaction\r\n-	The primer sequences should be place under 1 table.\r\n\r\nResults:\r\nMajor issues:\r\n-	What is F4 - F18? Are these refers to the 9 different isolates of Fusarium verticillioides? Why is it not named according to F1 - F9? I am guessing the FR200 and FR201 are the standard isolates. Why were the 3 non Fusarium verticillioides samples not included?\r\n-	The graph on MIC and MFC: is this a graphical representation of the table? If yes, then it is not required since the authors have presented in table form. In addition, what is the error bars refers to? Is it SEM or SD? This is not explained in the text nor in the figure legend.\r\n-	Both microscopic figures show different background colour, why? In addition, the figure for normal fungi was not sharp to appreciate the fungi morphology.\r\n-	The author have presented agarose gel picture of VERTF to differentiate the Fusarium verticillioides  from the non- Fusarium verticillioides isolates: isolates 3, 4 and 6 still possess a faint 400bp-band. In addition, there are also presence of additional bands (larger than 400bp) in isolate 2, 3, 4 6 and 7.\r\n-	The title of this manuscript is on the function of FUM1 gene, although the authors have included the FUM1 gene primers in the previous section, no results have been shown. \r\n\r\nCorrections:\r\n-	35 degrees Celsius > 35°C\r\n\r\nDiscussion:\r\nThis section needs major improvement to discuss about the results obtained.\r\n\r\nCorrections:\r\n-	spices ofFusarium > species\r\n\r\nCritical comments:\r\n\r\nThe English, formulation of certain sentences, and paragraphs need major improvement to provide readers better understandability of the manuscript.\r\nThe authors did not follow the Journal format . \r\nReferencing is not standardized in the manuscript.\r\n', 0),
(42039, 42049, 207, 0, '', '', 0),
(38209, 38219, 207, 0, '', '', 0),
(67403, 67416, 207, 0, '', '', 0),
(67719, 67733, 207, 0, '', '', 0),
(52823, 52835, 207, 0, '', '', 0),
(52823, 52835, 2524, 68044, '', '', 0),
(46442, 46452, 91, 68246, 'A good study but with many flaws and need to be reviewed thoroughly. It seem the authors just took it directly from his dissertation and send to the journal without following the journal requirement. ', 'You need to change form your dissertation format to journal format. Need major revision. Comment is attached in the reviewer form. ', 0),
(50703, 50713, 97, 67970, 'Abstract in Malay need to be revised.\r\n\r\nStatistics need to be verified as differences in percentages were large but found to be not significant.\r\n', 'Abstract in Malay need to be revised.\r\n\r\nDo not use abbreviation NNT in abstract.\r\n\r\nStatistics need to be verified as differences in percentages were large but found to be not significant.\r\n', 0),
(49270, 49280, 207, 0, '', '', 0),
(53295, 55059, 207, 0, '', '', 0),
(51980, 51992, 207, 0, '', '', 0),
(55156, 55169, 422, 68243, 'Reject due to methodology flaw', 'As stated in the form for each section', 0),
(54366, 54378, 2526, 68014, 'NA', 'The reviewer agree with the scientific merits of the study but the manuscript requires significant rewrite before it can be accepted. \r\nAbstract: (Malay): Please edit the objective to "kajian ini dilakukan untuk menentukan kesan sindrom metabolic aruhan air minuman fruktos ke atas ekspresi dan aktiviti dan enzim 11β-HSD1 di dalam hati tikus"\r\nAbstract: It was mentioned 12 rats were used in the Abstract but 18 rats were used in the Methods. Please check the numbers. \r\nIntroduction: Metabolic syndrome and Cushing''s Syndrome are two similar but unrelated conditions. I think the comparison is unnecessary here. \r\n...The activity of 11β-HSD in cell and tissue was first ''reported''... \r\nThe authors should acknowledge the original discoverers of 11β-HSD by citing the original papers, not reviews. \r\nThe authors should elaborate more on the role of 11β-HSD in metabolic syndrome, the research gaps in the field, and how they intended to bridge the gaps using this study. \r\nMethods: \r\n"Fresh fructose drinking water (FDW) was prepared every alternate day"... please edit. \r\nWas a particular region of the liver sampled since the enzyme was preferentially expressed near the blood vessels?\r\nWas the scoring performed by an anatomist or pathologist?\r\nPlease mention the manufacturer/developer, city, country of the antibodies, ELISA kit, ELISA reader, SPSS software used.\r\nThe manufacturer will usually mention the chemicals in each reagents. Please mention those chemicals instead of the "Reagent A/B" or "Substrate Solution"   \r\nLower case letter for "mean"\r\nMultiple pair-wise comparisons, not multiple group comparisons. \r\nResults: Please mention the p-value. \r\nDiscussion: \r\nParagraph 1 and 2 have been covered in the Introduction. They can be removed from the Discussion. \r\nThe authors should compare and contrast their findings with previous studies, even studies using a different model of MS. \r\nDelete "to utter surprise" since the findings are in line with the authors'' hypothesis. \r\nThe two paragraphs on how fructose drinking water can cause MS should be removed since parameters of MS were not measured in this study. \r\nPlease add the novelty, clinical importance and limitations of this study in the Discussions. \r\nConclusion: Please remove limitations from the conclusion paragraph. \r\nFigure 2: Please use a simple 2D bar instead of 3D bar', 0),
(38194, 38204, 466, 0, '', '', 0),
(65387, 65400, 113, 0, '', '', 0),
(67810, 67824, 2527, 68023, 'To further investigate whether the work has been published before or has been submitted elsewhere. I found the same abstract with a slightly different title appeared in this website, Int J Biol Med Res: http://www.biomedscidirect.com/2119//articlescategories\r\nI cannot find the full article nor the details of the volume no/page no. Maybe it has been accepted but yet to be assigned page no.\r\nIf it is being considered for publication elsewhere, it has to be rejected.', 'To elaborate on the methodology, results and strengthen the discussion.', 0),
(55156, 55169, 86, 0, '', '', 0),
(67534, 67548, 207, 0, '', '', 0),
(67736, 67750, 1886, 68132, 'A case report which lacks novelty. Accept if faced with shortage of articles', '', 0),
(54240, 54252, 233, 0, '', '', 0);

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  `pnid` int(10) NOT NULL DEFAULT '0',
  `decision` int(10) NOT NULL DEFAULT '0',
  `feedback` longtext NOT NULL,
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-- Dumping data for table `conference_decision`
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INSERT INTO `conference_decision` (`pnid`, `decision`, `feedback`) VALUES
(761, 2, 'test feedback'),
(903, 2, ''),
(907, 2, ''),
(956, 3, 'General:\r\n\r\nThe introduction ought to have highlighted the series of educational programmes as proposed by the Ministry of Health; it appears as though the educational lecture that was given is just a “one-off” attempt at providing CME to health care personnel and the main drawback of this paper is as though it was part of an “audit” /feedback of lecture content at the end of any „ordinary concept lecture‟. There isn‟t much new information that is derived based on this study – giving a “didactic‟ concept lecture as part of a CME is indeed widely practised but its effectiveness is always a concern. As such since the authors themselves raised the issue of having an interactive session, what new ideas or approaches can be incorporated to make the CME more effective? This would have added depth to the study. The results of doctors having better basic knowledge than paramedics is “reassuring” but this probably isn‟t surprising anyway – so conclusions derived at the end of this study probably does not add on to any “new” information.\r\n\r\nDetails:\r\n\r\n1. The introduction and review of literature was too generic.\r\n2. In the methods section, there was no mention of when the study was conducted and who were the actual sample population – ie was this carried out at a district/health or general hospital level as there will be variability in the respondents who attended. Also was it the same person who delivered the talk – this is important as the level of experience and knowledge of the speaker will influence the results of this study\r\n3. How was the content of questionnaire actually derived? Target group discussion or...? Mere extraction from the CPG is probably not the best way to go, esp if no additional discussions was carried out between relevant individuals. This needs to be\r\nhighlighted. The pretest was done on both doctors and supporting staff or...please clarify. Presumably it was delivered in English only or....? If so, will that have any restrictions (comprehension) on the paramedic personnel.....are nurses included in this group or...? Not entirely clear.\r\n4. Why was a cut off point of 1 week taken to analyse post K?\r\n5. How were these same 61 respondents captured again at post K level...?.please clarify.\r\n6. Concept of questionnaire validity is not correct.\r\n7. Results were not entirely clear – details of the questionnaire and scoring system used ought to have been highlighted further.\r\n8. When discussing results, it is the important facts or gaps in knowledge that ought to be highlighted rather than what was reported as high percentages (page 4 –last paragraph).\r\n9. Conclusion/recommendation – as mentioned, does not add on to any “new” information; maybe better to suggest if other CME modalities can be incorporated eg e-update/learning package on line as the study suggests that on-going CME helps with consolidation of core knowledge; so its these “innovative” suggestions that will add on „weight” to this manuscript\r\n10. References – not alphabetically arranged and name of journal ought to be italicised\r\n11. Style of writing really needs to be addressed. Suggest for a proof-reader to read and check the spelling and especially the grammar first prior to submission to any journal. Quite a significant amount of correction has been mentioned in the manuscript itself.'),
(900, 2, 'testing decision'),
(971, 2, 'This study generated some important data for corneal thickness and IOP for a local population.\r\nHowever, there are some methodologic flaws, which unfortunately, could not be salvaged at this point in time unless the study were to be repeated with the appropriate changes.\r\nThe greatest mistake was the tool used to measure one of the most important outcomes i.e. CCT was deemed unreliable because it did not have an inter-test variability data, rendering a major validity issue of the measurement outcomes. It is perhaps relevant to detail what this fault is so that non-ophthalmic people could further appreciate this limitation and make their own judgements about the final conclusions.\r\n\r\nTo “salvage” this study with some relevant info/output to the readership, suggest to analyse subgroups as follows:\r\n\r\n1) Emphasise this study was not powered to look at difference in aIOP because even though you have stat significant CCT decrease, the aIOP did not show a significant rise after HD.\r\n2) Look at the diabetics data (64% of cases) itself for homogeneity – do you see a trend worth discussing?\r\n    Also, how were the diabetic patients’ control – insulin??, their sugars?? Could this account for the fluctuation in the CCT or IOP results that generally do not agree with several other reports. \r\n3) Analyse separately the HD patients in the acute vs chronics i.e. <12 mths and >12 mths, which are of almost equal spread based on your numbers. The chronicity may be related to less fluid shifts during HD?\r\n4) Analyse according to the age group, look at the most commonly occurring age, this could remove any skewness of data because of age-related fluid shifts, if present.\r\n\r\nOtherwise, the following are some minor errors that need to be addressed and the appropriate amendments are required:\r\n\r\nAbstract – the methods section -  statistical tests and measures need not be included\r\n                 Results - ….. differences in IOP before and after HD…\r\n                Conclusion -  when correcting for the adjusted IOP…. (Not …when measuring the IOP at least)\r\n                Please provide an unstructured abstract of 250 words without subheadings in both Malay and English language with a list of three to six keywords.\r\n\r\nIntroduction:\r\nSpell out what CCT is in full, central corneal thickness, at least once in the beginning.\r\nSeventh line from the bottom, para 2 :   Haemodialysis results in…..\r\n\r\nMethods\r\nPara 1, second sentence from bottom:  …3 days a week….\r\n\r\nResults\r\nPara 1, Females “outnumbered” males – it’s not a competition, re-phrase : There were more females than males and the majority were diabetics (64%).\r\nLast para, second sentence from bottom.  Although the corneal thickness…… in 36% of eyes –  OMIT because it’s self-explanatory by statement earlier that 64% decreased in thickness.\r\n\r\nDiscussion\r\n3rd page:  In our study, the largest (not highest) rise……  and the greatest (not highest) decrease\r\n                4th sentence from bottom, speling error -  hypothesized\r\n4th page:  Para 1 -  about the CCT and correlation.  What other factors might play a role??? Elaborate, please. How about your study sample size, which is very small, the CCT measurement which is questionable and also the heterogeneity of the cases, which include diabetics, young and old, early unstable and chronic stable HDs ??\r\n5th page: Para 3 -  another limitation of this study…..  not intra-observer bias BUT a potential systematic error in measurement.\r\nLast para:  …..in a skewed high risk population in UKM MC…  (drop … Malaysian patients  …because it’s not representative).\r\n\r\nFigures: Remove all correlation figures 1-4 – non-significant, so just describe in the text these results in the RESULTS section.\r\nTable 1: Age range is actually very wide – 23-77,  you should use median instead of mean age.\r\nTable 3: Footnote:  rephrase to *statistically significant on paired t-test.\r\n'),
(946, 2, 'testing190813'),
(979, 2, '1.testing\r\n2.testing\r\n3.testing'),
(968, 2, 'This worked is still worth to be published but major review need to be done. My advices to the author are:\r\n\r\n1.	Focus what important or main message you want to get across and then write with more depth about it.\r\n\r\n2.	Improved the English used in the article.\r\n\r\n3.	Avoid writing the abbreviation first follows by the full sentences but instead vice versa.\r\n\r\nSpecific comments:\r\n\r\nOn Abstract: Should include what are the events or complications that happened post injury in the abstract. The first two sentences can be shortened and make it as one sentence. Author should make full used of the space given in the abstract for informing most of the issuethey want to highlight.\r\n\r\nThe abstract written in Malay was not well translated leading to confusion in the meaning. A better way to right the abstract in Malays given in this example, Kejadian keganasan seringkali dianggap sinonim dengan lumrah kehidupan dikebanyakan bandaraya diseluruh dunia. Keganasan yang terjadi seringkali mengakibatkan pelbagai jenis kecederaan termasuk kecederaan tusukan pada dada. Kecederaan ini boleh disebabkan oleh kemalangan jalanraya atau lika tikaman yang terjadi akibat kes pembunuhan ataupun cubaan buuh diri.......’\r\n\r\nOn Introduction: Do not use abbreviation or acronym followed by the full/expanded meaning of the abbreviation/acronym. It should be the other way around.\r\n\r\nOn Case Report: \r\n\r\n1. The sentences phrasing need to be improved.\r\n\r\n2. Figures need to be numbered\r\n\r\n3. The English need to be improved especially in sentences phrasing. Noted some part of the article contained a lay person spoken English. E.g. Initial blood pressure was on lower side 80/40mmHg.........’ instead the author should just write ‘Initial blood pressure was 80/40mmHg,’	\r\n\r\n4. Paragraphs 6 last 2 sentences has no correlation. Was there a planned to admit this patient  and then the patient refused and took at-own-risk discharge? Because it was never mention prior to that sentence.\r\n\r\n5. Avoid using abbreviation without writing the full meaning of it first. Eg MOPD, LV, RA/RV.\r\n\r\nOn Discussion: \r\n\r\nThe discussion was not focus towards the topic which is “Anterior penetring chest injury complicated with delayed left ventricular thrombus embolization”. I think the first three paragraph sway off the focus of this case. I think it shoulds be removed. Further elaboration on the complications of Anterior Penetrating stab injury with of LV thrombus would be good. More searches should be done in this area.\r\n'),
(961, 2, ''),
(972, 3, ''),
(1067, 2, 'Review comments:\r\n\r\nAbstract:\r\n1. The introduction should emphasize on the role of screening of thalassaemia in blood donors rather than introduction on thalassaemia in general.\r\n2. The total number of donors screened did not tally to the total of numbers based on the ethnic distributions. \r\n3. Mentioned in abstract there were16 Hb E traits. This did not tally with the number shown in Table where n=19.\r\n4. No need to underline the keywords.\r\n\r\nIntroduction:\r\nThe last sentence in your introduction is more suitable in writing for research proposal. Now you are publishing the work therefore the info is not relevant.\r\n\r\nMaterials and methods:\r\n1. Need to explain the criterias for selection.\r\n2. The name of the company/ies is/are required in detail.\r\n3. ‘acceptable haemoglobin’ for what purpose? Must mention.\r\n\r\nOthers:\r\nThe results and discussions are quite fair comprehensive. However, the author/researchers should attempt to confirm the findings with DNA study for possible Alfa thalassaemia. The conclusions however, did not summarize the issue regarding title of this write up rather, the author talked about the use of Hemogram in this study. Thus, further reconstruction of the text required.\r\n\r\nThe discussion regarding the diagnosing of iron deficiency in this study is also very confusing as it is not part of the research/write up.\r\n\r\nIt is also noticed that the author did not use any statistical analysis to analyse the data.'),
(1144, 2, '\r\nGood effort in producing this article. A few comments:\r\n\r\n1.	There is no Figure 1 & 2 attached.\r\n2.	The abstract in Malay does not mirror the abstract in English. Please revise the wordings to reflect the exact message in the English abstract\r\n3.	Please provide keywords in Malay.\r\n4.	Page 8. “This effort of resuscitation may be temporarily acted on static lactate, as it had slowed down after the achieving a lower static lactate level at H1.” Please correct the grammar of this sentence.\r\n5.	Page 9. “Our study may introduce a selection bias that was difficult to account for, as there were plights to track patient’s whereabouts in the first 6 hours in the hospital.” Please correct the sentence.\r\n6.	Table 2. Why are organ severity, sepsis and mortality higher in survivors compared to non-survivors?\r\n7. 	Please provide fax and telephone numbers for the corresponding author.\r\n'),
(1137, 2, '1. Comments has been incorporated into the paper itself.\r\n\r\n2. Many deficiencies but has good potential.\r\n\r\n3. Some parts of the manuscript are very similar to earlier published literature. Please rephrase these sentences because it may lead to plagiarism and unnecessarily publishing conflicts.\r\n'),
(1152, 1, '1. Introduction section is missing. Please add few sentences with 1-2 references under the subheading of "Introduction".\r\n\r\n2. Discussion: Please follow the style of referencing needed for Medicine & Health.'),
(1282, 2, 'The manuscript has been well written. Please begin the abstract with a background sentence. Please add limitations of study. How many observers took part? How was error minimized? Please add few latest references from 2013/2014/2015.'),
(1261, 2, 'The following clarifications need to be addressed and revised;\r\n\r\nAbstract:\r\n\r\n1.  The study consisted of one group of participants and there is no control group involved.  Why?\r\n2.  As the study is having only one group, why Independent sample t test was used?  Why not Paired t-test?  The given table also indicated independent sample t test has been used?  The statistical test chosen is inappropriate.  Please revise.\r\n\r\nIntroduction:\r\nSatisfactory\r\n\r\nMethods:\r\n1. Why quasi experimental study design was chosen for this study?\r\n2.  Self reported Functional Ankle Instability?  kindly add the validity of FAI in clinical context to strengthen the script\r\n3.  There are different types of ankle injuries which varies from fracture, muscle strain, unilateral or multiple ligament failure of different grades.  Therefore, please specify what type of ankle injury are you talking about\r\n\r\nResults:\r\nPlease add the demographic characteristics of the subjects\r\n\r\nDiscussion:\r\n1.Add clinical implications to enhance the strength of the work\r\n2.You have stated future study required with increased sample size?  How did you calculate the sample size of the current study and what is the deficit?\r\n\r\nConclusion:\r\nConsider tone down the conclusion\r\n'),
(1293, 2, '1. Title page is incomplete. Please provide full names of all authors, author’s affiliation, corresponding author’s complete postal address, telephone and fax number, and email address.\r\n\r\n2. The short running title is missing.\r\n\r\n3. Please add one or two sentences on the background. \r\n\r\n4. The last sentence of the abstract should explain what is the new scientific fact while reporting this case. Please do not write that manifestation will be discussed.\r\n\r\n5. Introduction: Please submit the document with evidence of obtaining permission to reproduce figure 1A. We need it for official purpose.\r\n\r\n6. References at the end should have issue number which was found to be missing. Please revise and resubmit.\r\n'),
(1707, 2, '1. Title Page:\r\n\r\nTitle page must include:\r\n•	Institutional affiliation- address(es) from the work originated linked to authors’ names by superscript\r\n•	A short running title\r\n•	Email, telephone and fax number of the corresponding author.\r\n\r\n2. Format:\r\nThere should be no section on “Literature Review”. Please combine with “introduction”.\r\n\r\n3. Abstract\r\nThe statement “Mesin ultrasound di gunakan untuk mengenalpasti cecair di bahagian abdomen, cecair di dalam lapisan luar jantung, angin di dalam rongga dada, dan keadaan hidrasi pesakit” in BM abstract doses not appear in English Abstract. Suggest to remove.\r\n\r\nConclusion of BM and English abstract does not match. Please recheck.\r\n\r\n4. Keywords\r\nPlease include a list of three to six keywords in both Malay and English from MeSH database.\r\n\r\n5. Introduction\r\n\r\nEFAST = give full name and short description \r\n\r\nPlease follow format: the objectives and the methodology should be in sentences form. No bullets or lists allowed.\r\nPlease provide ethical approval code/no .\r\n\r\n6. Tables\r\n\r\nTables should be placed in the main manuscript file at the end of the document, not within the main text.\r\n\r\nEach table should be cited in text.\r\n\r\nTable 2– better to arrange the Malaysia races followed below by foreigners\r\n\r\nTable 3 Diagnostic test – standardize the figures to 2 decimal points.\r\n\r\n7. References\r\n…..by Fenkl (as cited in ed. Adam Brooks Et al. 2004), \r\nBitschnau (as cited in ed. Adam Brooks Et al. 2004).....    Please cite the reference for Fenkl and Bitschnau\r\n\r\nErrors in the citation eg Corinne Chmiel  Et al. 2011 should be Chmiel et al. 2011\r\n\r\nComplete author citation is required for References section (use of et al is not acceptable).\r\n\r\nCheck back the format of the references so that they comply with the author’s guideline.\r\n\r\nMinor correction:\r\n\r\n‘keratan rentas – cross sectional)  - no bracket \r\nogos – capital letter “O”\r\n'),
(6026, 2, 'The manuscript was submitted in the form of a dissertation and not in the form of a journal manuscript. Obviously you did not read and follow the ‘Guidelines for Authors’\r\nYou need to re-format the whole article into the form of a journal manuscript and resubmit again.  \r\n\r\nRefer to ‘Guidelines for Authors’\r\n\r\nTitle Page:\r\nTitle page must include:\r\n•	A short running title\r\n•	Email, telephone and fax number of the corresponding author.\r\n\r\nKeywords	\r\nNo keywords provided. Please include a list of three to six keywords in both Malay and English from MeSH database.\r\n\r\nIntroduction\r\n-	Too scanty, need to elaborate the problem further worldwide and Malaysia.\r\n-	No references were given for all the data presented. \r\nE.g \r\n“The numbers of early revisit among adults with AEBA discharged from the ED is significant- what was your figures? Any reference to support.\r\n-	The objective was written in the last para of the introduction repeated in another section. This is not the way to write a journal manuscript. \r\n\r\nMaterial and methods\r\n-	No need to show how you calculate sample size.\r\n-	Study design mention not correct. \r\n-	All should be in a text form. \r\n-	Research & Ethical clearance reference number should be stated. \r\n-	Study flow put in appendix if need be, if not can remove it. \r\n-	Refer Guidelines for Authors’\r\n\r\nResults\r\n-	Just report the result, please. Do not give your comment or opinion or quote other study reference in this section. \r\n-	Too many table / figures. Please choose the important results you want to highlight. Need to separate the figure and tables from the main text (Refer to Guideline to authors)  \r\n\r\nDiscussion\r\n-	Please state the limitation to this study. \r\n\r\nReferences – Not follow the guidelines given.\r\n'),
(7236, 2, 'The study concerns a linguistic validation on HypoSRQ, a questionnaire to detect hypoglycaemia.\r\nAbstract: The BM abstract was very different from English version. Please translate the English abstract directly into BM.\r\n\r\nOverall, a good manuscript with detailed methodology to validate the HypoSRQ questionnaire. A few grammatical errors that require proof reading.\r\n\r\nReferences: please check back the format.\r\nCannot find these authors Hans TS, Ward H, McBride J, Yap I 2013,  in the reference lists. \r\n\r\nConclusion: suggest to be direct and only 1 to 2 sentences in length.\r\n'),
(7244, 2, 'Review comments 1:\r\n\r\nGeneral: \r\n1.	The paper focused on the details of Weibel technique in bone histomorphometry quantification. Unlike other original articles, this paper described a technique thus the conventional IMRAD is not present. This paper can be the reference to others who will be doing the same analysis.\r\n2.	Several typo errors are observed eg ‘Histomorphometric’ in the title itself, ‘Weibel’ in the running title. There are other errors throughout the paper. Grammatical errors are also observed. Authors to scrutinise the paper thoroughly for amendments.\r\n\r\nTitle:\r\n1.	I suggest adding ‘In animal model’ to the title. \r\nIntroduction\r\n1.	Page 2, last para: Authors mentioned a list of studies which used Weibel technique for bone histomorphometry. And the technique was not described. However, these studies must have referred to other studies in performing the protocol. Please elaborate further on how referencing was done in other studies. They referred to several sources? What are the sources? So this paper is written to compile the whole technique in one paper? Please elaborate to clearly justify the purpose of this study.\r\n\r\nMethodology\r\n1.	Please specify the species of animals used. How many femurs/rats were used? In the flow chart (Figures 1 & 2) left femur was stated but in the Methodology, it was not mentioned. Any ethical approval?\r\n2.	Figure 1 did not tally with the first paragraph in Methodology. In the paragraph, bone was fixed in 70% alcohol after which bone was cut sagittally. But in figure 1, the flow was cut sagittally then fixed in alcohol. Please revise.\r\n3.	Figure 2: there are also some discrepancies between Figure 2 and the text page 4 and 5. \r\n4.	Page 5: Please put a sentence eg The decalcification process is summarised in Figure 2. Because the way it is now, Figure 2 was written after haematoxylin and eosin sentence, which may confuse the reader that Figure 2 may be refering to the staining process.\r\n5.	Figure 3: Again, the text on procedure and the fact on Figure 3 did not tally. Please revise.\r\n6.	Figure 4: same comments as above. Procedure page 7, second para: In figure 4, embedding was for 48 hours but not mentione in the text on page 7. Figure 4 also stated von Kossa staining but it was not described in the text procedure.\r\n\r\nDiscussion\r\n1.	Discussion too briefPage 13. Authors mentioned that there are several errors that might lead to false results. From where did the authors derive this? Is there any papers discussing on possible errors which authors referred to?  It will be good if the arguments are supported by other literature.\r\n\r\nReferences:\r\n1.	Please revise the format according to the journal’s guidelines.\r\n\r\nReview comments 2:\r\n\r\nThe manuscript is well written. Detailed technique was mentioned in the text. The methodology was comprehensive. However, the authors did not focus on the discussion section such as \r\n\r\n1. Justification of using this technique\r\n2. Discussion on the results achieved.\r\n3. Comparison of the technique with the other standard routine procedure\r\n4. What are the benefits and limitations of using such technique (in detailed analysis).\r\n\r\nA few spelling mistakes are noted in the text as well. \r\n\r\nSome of the section on Methodology such as H&E staining, need to be less comprehensive as this staining is the standard histological staining. \r\n\r\nThe authors need to provide the strong justification of using Weibel Technique to support the findings.\r\n\r\n'),
(13155, 2, '1. Title & Abstract:  The title is too long. May shorten to : Retinal Nerve Fiber Layer Thickness Post-Laser Treatment in Diabetic Retinopathy: Argon versus Pattern Scanning Laser. The abstract should be unstructured with no subheadings. \r\n\r\n2. Introduction: well written\r\n\r\n3. Material & Methods/Case: This section should ideally have subheadings. There are certain grammatical errors: for eg. “criterias” , instead of criteria.\r\n\r\n4. Results: As the results have been tabulated, authors need not mention all findings. Only the significant/ important findings should be highlighted. \r\n\r\n5. Discussion: the first 2 paragraphs sound  too general. The authors should stay focused and discuss about this study findings. \r\n\r\n6. The tables are rather untidy. Continuous variables should be presented as either mean ±SD or median (range).\r\n\r\n7. Please submit the figures in JPEG files. Figures ‘pasted’ into Word files are not accepted.\r\n'),
(27848, 2, 'Review comments 1:\r\n\r\nThe study is interesting but few facts need to be addressed. \r\nPlease add limitations of the study. \r\nPlease add recommendations, if any.\r\nRegarding the references at the end, please do not write the name of the journal in italics. Please write the issue number for each article. \r\nPlease reduce the figures. You may have maximum of 10 figures. \r\n\r\nReview comments 2:\r\n\r\nThis manuscript reports the protective and therapeutic effects of three different herbs in TNF-α induced inflammation in hypothalamic GT1-7 cell line. \r\n\r\nTitle & Abstract: \r\n- The title should avoid the use of non-standard abbreviations, e.g. (L.) DC. Var. \r\n\r\nIntroduction: \r\n- In paragraph 1, the author(s) should provide some statistics for the aged population in recent years, instead of just mentioning the high rate (21.1%) in 2050. \r\n\r\nMaterial & Methods/Case: \r\n- What is the concentration of DMSO used to dissolve the plant extract? Is it 0.1% DMSO as used as a standard in MTT assay? \r\n- Suggest to lists the primers as a separate table. \r\n- in the western blot section, 1 hour of TNF-α exposure was not mentioned, it was only mentioned in the discussion. \r\n\r\nResults: \r\n- The data was represented as SEM (as mentioned in materials & methods section) or SD (as mentioned in figure legends)? \r\n- TNF-α induced apoptosis, cytotoxicity or inflammation? \r\n- Please provide justification why both Z. montanum and T. laurifolia were prepared as ethanol extracts while G. pseudochina were prepared as methanol extracts. \r\n- G. pseudochina leaves [hexane extract (GE)]: should it be GH? \r\n- Why GT1-7 cells were exposed to TNF-α 3 or 24 hrs? \r\n- In Fig 1 and 2, the untreated, TNF- α treated and 0.1% DMSO treated should be presented as a single bar only. \r\n- Please delete the lines “The experiments were designed to evaluate both protective and therapeutic effects. For their protective effect, cells were pretreated with selected concentrations of extracts for 24 hours and then were incubated with TNF-α (10 ng/mL) for 3 hours. For their therapeutic effect, cells were incubated with TNF-α (10 ng/mL) for 3 hours and then were treated with selected concentrations of extracts for 24 hours.”\r\n\r\n- Fig 3G was not provided. \r\n\r\nDiscussion: \r\n- “Our present results further support the neuroinflammatory mechanism via NF-κB signaling pathway that exposure to TNF-α for 1 hour …..” 1 or 3 hours?\r\n\r\nFigure legends:\r\nRefer to the legend of Fig.1, Fig.7 and Fig 8. Is it mean ± SD or SEM?\r\n\r\nCritical comments: \r\n- All abbreviations used should be defined in the text where it is first used: TNF-α, IL-1β, IL-6, iNOS, NF-κB and IKK-β in paragraph 2 were not defined. \r\n- English needs to be improved in the manuscript, especially spacing between words. \r\n- In reference section, scientific name for herbal plants in the journal title need to be italic.\r\n'),
(27870, 2, 'Review comments 1:\r\n\r\nPlease submit the revised version of the manuscript in MS Word (97-2003) file.\r\nThe running title should have few words, only. At present, the running title is too long. \r\nPlease insert page number on each page.\r\nThere is no need to highlight the name ‘figure’ in 3.2.\r\nIn section 3.5, please 1303.09 μg Kg-1 as 1309.09 μg/Kg\r\nPlease add few limitations of the study.\r\nPlease rectify the style of referencing at the end section. An example is shown for the first reference. Please also follow PubMed for the correct representation of surname and name. The issue number has to be added after volume. An example is- Lewy, A.J., Bauer, V.K., Cutler, N.L., Sack, R.L. 1998. Melatonin treatment of winter depression: a pilot study. Psychiatry Res 77(1):57-61\r\n\r\nReview comments 2:\r\n\r\nTitle & Abstract:\r\nThe title and abstract are appropriate. The running title is too long, please shorten it. \r\n\r\nIntroduction:\r\nThe introduction covers sufficient background of the study. Some references dated 1999 need to be updated.\r\n\r\nMaterial & Methods/Case:\r\nThe materials and methods are described in sufficient details. I only have two comments here: \r\n1.	Are the active compounds (tryptophan and melatonin) stable at 70 degree Celsius for 30 mins?\r\n2.	Please mention the manufacturer, state and country for SPSS version 17. \r\n\r\nResults:  \r\nThe results are very comprehensive and described in a logical manner. I have a few comments: \r\n1.	“But brown rice extracts with water at high concentration…decreased cell viability” The effects were not significant compared to hydrogen peroxide treated cells. It is at best a trend. I suggest to delete the statement or mention it was just a trend but not statistically significant. \r\n2.	“Quercitin (spelling error? Do you mean quercetin?) was used as sample control” Please explain why quercetin was used as positive control, rather than standard antioxidant like alpha-tocopherol, or the postulated active compounds tryptophan and melatonin. \r\n3.	Can you explain why water extracts of black glutinous rice and baby corn contain the highest amount of tryptophan but it was ethanol extracts for other plants?\r\n4.	Can you explain the following discrepancies: \r\na.	Water extracts of black glutinous rice and baby corn have the highest tryptophan but their ethanol extracts have the highest antioxidant activity. \r\nb.	DPPH results were better for hexane extract of sweet corn, but its ethanol extract has the highest amount of tryptophan and melatonin. \r\n5.	For the figures, please mention that the p<0.05 or <0.01 is between the indicated group and the group treated with hydrogen peroxide only. \r\n6.	Please provide list of abbreviation for all figures. \r\n7.	Figure 2: any statistical significance between the groups?\r\n\r\nDiscussion:\r\nPlease divide the discussion into appropriate paragraphs. Please indicate the limitations, novelty and clinical implication of this study in the discussion. The conclusion is sufficient. \r\n\r\nCritical comments:\r\nPlease revise the language and spelling of the paper.\r\n\r\n'),
(25501, 2, 'Review comments 1:\r\n\r\nIntroduction: \r\nToo long. Need to be shortened and should be concise. \r\n\r\nThe references cited in this manuscript are not from recent publications (later than 2011). Almost all are quite old references. \r\n\r\nMaterial & Methods/Case: \r\nThe calculation for sensitivity of VRPI to detect those with CVD risk factor in the ‘Statistic analysis’ should be put elsewhere in the Methods, not under this subtitle. \r\n\r\nResults: \r\nIn the Methodology, it was stated that the sample size was 24 per group (24 x 3 = 72), but in the Results, it was mentioned that the total patients involved were 114. Any drop-out? \r\n\r\nDescriptions of results were not clear. Did not indicate the increase was between what groups. The p values in the Tables 3 and 4 indicate what? Comparison between what groups? ANOVA only tells if there is any difference amongst the groups but does not indicate between what groups. Did you run post-hoc test to determine which groups were significantly different? Did you run normality test? \r\n\r\nPlease replace ‘association’ with ‘correlation’ in Tables 5 and 6.\r\n\r\nDiscussion:\r\nIn the Discussion, any reference to tables or figures in the Results should be avoided.\r\n\r\nPlease replace ‘association’ with ‘correlation’ because they mean differently.\r\n\r\nAlmost all references are old.\r\n\r\nAny suggestion should not be in the Conclusion. Put suggestion in the Discussion.\r\n\r\nPlease check for some grammatical errors in the manuscript.\r\n\r\n\r\nReview comments 2:\r\n\r\nTitle & Abstract:\r\nAn easily-comprehensible title with a concise abstracts.\r\nThe keywords need to be revised. Please include 3-5 keywords from MeSH database. \r\n\r\nIntroduction:\r\nInformative-the authors have introduced the necessary terms and explained the significance of this study by mentioning the association between PPGF and several other markers of CVD\r\n\r\nMaterial & Methods/Case:\r\n\r\nThe authors should clarify further on how VRPI was determined (page 8 – method’s section). Table 1 showed the different classification of VRPI (very low risk/low risk/moderate risk/high risk) based on the risk scoring provided – however it was not mentioned how this risk scores were derived. Was it based on the presence of the CVD risk factors for eg systolic or diastolic blood pressure (BP) ≥ 140/90 mmHg or on antihypertensive medication, abdominal obesity (waist circumference >90 cm), smoker, dyslipidemia (Total cholesterol >6.2 mmol/L or low density lipoprotein >4.1 mmol/L or triglyceride >1.7 mmol/L or high density lipoprotein <1.02 mmol/L) or family history of premature CVD as mentioned? Please clarify.\r\n\r\nResults:\r\n\r\nResults were outlined systematically. However for table 4, it was not clear why the results for some of the vascular properties were adjusted for the age, race, HR and height while some were not? Were these parameters known as confounders for specific vascular properties? Please clarify.\r\n\r\nDiscussion: \r\nWas there any other study which recruited participants with a relatively younger age group in accessing the usefulness of PPGF in detecting CVD risk? The authors did mention studies assess CVD risk in older age group, however these participants presented with quite advanced vascular disease progression. It would be more valid to compare with studies which recruited participants with similar age groups.\r\n\r\nCritical comments:\r\nThis study evaluated the use of PPGF in assessing younger participants with CVD risk factors. The objectives were clear, and the methodology was outlined reasonable well. Results were also outlined systematically. However, this manuscript would benefit from some minor clarifications/revisions (as mentioned above).\r\n\r\n'),
(7354, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\n‘Expression of p53 protein in premenopausal age women as risk factors for breast cancer ‘. ‘age’ can be removed to shorten the title. Capitalise each word.\r\n\r\nAbstrak: …. oleh (Immunohistochemistry)IHC...’IHC’ should be the one in bracket.\r\n.... kategori proteein....correct the spelling.\r\n\r\nAbstract: Please standardise the way the value is presented. Add 0 before the decimal eg 0.08, 0.52…. Standardise to 2 decimal place only.\r\n\r\nIntroduction:\r\nLots of grammatical errors that requires checking.\r\nPlease use past tense.\r\nPlease use the abbreviation once it has been defined eg IHC. Do not repeat the full term.\r\nAmong them:\r\nIn normal cell cycles (add a comma) of proliferation and regression are controlled by….\r\nA Malaysian women’s cumulative risk of breast cancer in her lifetime is in the ratio of (1:20) with the highest risk of occurring in Chinese women (1:14)\r\n\r\nMaterial & Methods/Case:\r\nPlease use past tense to describe the methodology.\r\nLast paragraph – incomplete sentence: ‘See Figure 1 for’\r\n\r\nResults:\r\nStyle of presenting data could be improved. Limit to 2 decimal place. Add 0 before decimal.\r\n\r\nConflicting statement:\r\n‘In addition, majority of the respondents aged 41 years and less also have positive p53 (+) protein category (n= 3/111)’. Should be minority n=3\r\n\r\nDiscussion\r\nGood discussion but need to check the grammar.\r\n\r\nConclusion\r\nToo long. Limitations of the study in the second paragraph should be moved to Discussion section.\r\n\r\nFigures and Tables\r\nStandardise the values: Limit to 2 decimal place. Add 0 before decimal.\r\nThe photomicrograph is in black and white. Cannot appreciate the be brownish precipitations.\r\nWhy is the magnification not standardized. X10 or X40\r\nWhat is S0045464 and other similar labels? Please mention them in the footnote of the Table.\r\n\r\nCritical comments:\r\nthe manuscript requires grammar corrections by an expert in English.'),
(29410, 2, 'Review comments:\r\n\r\nPlease separate the words jumbled in the title.\r\nThe keywords need to be revised. Please include keywords from MeSH database.\r\nIn references, journal names should be abbreviated according to the Pubmed/Medline.\r\nPlease send the copyright declaration form in case you have not sent it earlier.\r\n'),
(29692, 2, 'Review comments:\r\n\r\nPlease write the abstract as follows:Start the abstract with few sentences on the background. Then only, highlight the case. The ending portion of the abstract should clearly state the clinical implication/significance for the case.\r\n\r\nSome keywords need to be revised. Please include keywords from MeSH database.\r\n\r\nPlease write the references conforming to the journal style. The journal does not accept references with numbers. \r\n\r\nReferences in the text should contain the authors’ names followed by the year of publication without punctuation mark. Please refer to recent published articles.\r\n\r\nReferences should begin on a new page, be double-spaced and listed according to alphabetical order.\r\n\r\nPlease revise the English language in the text. There are many sentences which are too long and grammatically incorrect. Please shorten these sentences.\r\n\r\nPlease revise the manuscript accordingly. \r\n\r\nPlease submit the Declaration Form which has to be signed by all authors and the scanned copy has to be e-mailed to the Medicine & Health address. In case, the authors fail to submit, the article will not be published. Declaration Form is a declaration on the part of authors and we weigh it seriously because of legal aspects which may take place, later on.'),
(29959, 2, 'Review comments:\r\n\r\nMany grammatical errors . Require proofreading by person with good English.\r\n\r\n1. Abstrak BM\r\n\r\nMake sure the BM abstract is translated correctly\r\n- Congenital Nasal Pyriform Aperture Stenosis = stenosis bukaan piriform nasal kongenital\r\n- …(CNPAS) adalah penyakit yang amat jarang menyebabkan kesempitan saluran udara hidung\r\n- Secara klinikal, ia seperti atresia choanal pada bayi neonat.\r\n- Walaupun Sedikit kesempitan di kawasan keratan rentas hidung, ia akan meningkatkan rintangan saluran udara hidung menyebabkan hidung tersumbat.\r\n\r\nAmong the grammar corrections are:\r\n\r\n2. Abstract:\r\nThus nasal obstruction can be easily occur if there is slight decrease in its cross sectional area.\r\n\r\n3. Introduction:\r\n- Neonatal nasal obstruction rarely caused by Congenital Nasal Pyriform Aperture Stenosis (CNPAS).\r\nOne of the rare cause of neonatal nasal obstruction is Congenital Nasal Pyriform Aperture Stenosis (CNPAS).\r\n- In order to assess the level of obstruction, thin axial sections through the midfacial region in is needed.\r\n- born via emergency caeserean section\r\n\r\n4. Discussion:\r\n- The incidence It was estimated to be about 1 : 25000 to 1 : 50000 births (Wieniewska et al. 2002).\r\n- However, in the present case, the infant have has no cleft lip or palate.\r\n- Arlis and Ward has reported four out of six patients with CNPAS has presence of SCMI.\r\n- Neither holoprosencephaly nor single central maxillary incisor are is a common feature of Trisomy 8 Mosaicsm.\r\n\r\n5. Other queries:\r\n\r\n- Introduction: we report the first case of CNPAS……the first case in Malaysia? Kuala Lumpur?\r\n- Discussion: The latter can be a part of CHARGE syndrome (Al Abri et al. 2008)- Please give the full term for CHARGE.\r\n- Any acknowledgement?\r\n\r\nOverall, an interesting case-report. The manuscript requires grammar corrections'),
(27866, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\n\r\nSolar Retinopathy with Interesting Imaging Findings\r\n\r\nPlease endeavor to perform a few other investigations to confirm it is not due to other pathology. \r\n\r\nSeveral errors in the abstract:\r\n\r\nSolar retinopathy is an irreversible injury of the retinal photoreceptors due to direct viewing of the sun. not true as several articles say direct viewing is not always  the case. Sun/ light exposure is all that is necessary. Diagnosis of the disease is challenging and requires combination of a detailed history and imaging modalities. You need to show that this is indeed solar retinopathy. This case report focuses on a 55-year-old fruit picker with an irreversible central scotoma of the right eye. A diagnosis of solar retinopathy was made based on history but mainly by several imaging modalities, such as optical coherence tomography (OCT), infrared (IF) imaging of the fundus and fundus autofluorescence (FAF). In conclusion solar retinopathy is an irreversible disease where multimodal imaging may play an important role in the diagnosis. The condition is irreversible thus advocating protective eyewear is mandatory in patients who are chronically exposed to sun. Not true that all are irreversible. Patients recover over a number of months unless the damage is very severe.\r\n\r\nNot sure why eclipse is a keyword\r\n\r\nIntroduction:\r\n\r\nOK\r\n\r\nMaterial & Methods/Case:\r\n\r\nResults:  \r\nSD-OCT showed generalised outer retina thinning of the right macula, with an intact external limiting membrane (ELM). There was presence of a sudden disruption of inner and outer segments of photoreceptors at the fovea with cavitations. Segmentations of retinal pigment epithelium (RPE) interdigitations were noted. Otherwise, the RPE/Bruch’s membrane complex was intact. (Figure 2)\r\nThe ELM does not really show intact layer. There is some subtle discontinuation also.\r\nWe proceeded by performing IR and FAF tests of the right eye. IR revealed multiple hyper-reflective areas at the macula region. Look more hypo than hyper. (Figure 3)FAF showed a rim of hypoautofluorescence surrounding the fovea with intervening area of hyperautofluorescence. (Figure 4) The picture actually does not show this clearly. The OCT, IR and FAF of the left eye appeared normal. In view of his exposure to sunlight and findings from the OCT, IR and FAF, a diagnosis of right eye solar retinopathy was made. \r\n\r\nTo this date, this patient’s right vision maintained at 6/36 with no improvement with refraction. As solar retinopathy is an irreversible condition, we counselled him that his right vision may not improve in future and advised him to use protective eyewear to protect his left eye. \r\nThe actual duration of the follow-up needs to be stated. It should be long enough to show irreversibility of the condition despite reduction of sun exposure.\r\nPlease discuss the role of FFA and mfERG in excluding other condition such as mac tel and how it should differ in this patient. Because we want to show this is truly solar retinopathy and not something which mimics it.\r\n\r\nDiscussion:\r\n\r\nSolar retinopathy is a disease of the outer retina layer. Although the disease commonly affects both eyes, there have been previous reported cases of unilateral solar retinopathy such as our patient. (Sheth Jay et al. 2013, SP Dhir et al. 1981) It is postulated that unilateral cases are attributed to the uneven reflex blinking of the eyes and one eye being the more dominant than the other, hence the dominant eye is affected more. \r\nThis patient’s clinical examination also does not show a red spot which is pathognomonic for solar retinopathy. Explain why this differs in this patient.\r\n\r\nIn our case, OCT showed generalized thinning of the outer retinal layers with preservation of the inner retinal layers thickness. This is in keeping with the fundamental understanding that solar retinopathy manifests as an outer retinal pathology. Interestingly in our case, the ELM layer was preserved. Why? Another interesting feature seen in the OCT of this case is the presence of cavitations at the photoreceptor layers, similar with the cavitation at the inner retinal layer seen in macular telangiectasia type 2. (Charbel Issa P 2013) We postulate that this is most likely due to the loss of the photoreceptor tissues.\r\n\r\nCritical comments:\r\nThe case is nice but because it is trying to show unusual imaging findings, we have to be very certain this is solar retinopathy indeed. Hence the other investigations to confirm. Because on clinical picture it does not look typical. \r\n'),
(28918, 2, 'Review comments:\r\n\r\nPlease avoid using the name of the institution in the title. You can simply write your years of experience.\r\n\r\nRectify the running title. \r\n\r\nIn the abstract, please write the full name of UKMMC not National University.\r\n\r\nReduce the word count of the abstract to less than 250 as this is the journal requirement.\r\n\r\nMaximum 3-5 keywords from MeSH database are needed.\r\n\r\nPlease refrain from writing ‘we’ every time in the text. Rephrase the sentences with ‘we’.\r\n\r\nPlease do not try to mention/explain facts in the methods section.\r\n\r\nPlease give a separate subheading for statistical analysis. \r\n\r\nPlease highlight the exclusion criteria as well.\r\n\r\nIn the results section, refer to the first line, you cannot start a sentence with numerical. Rectify it.\r\n\r\nWrite the manuscript in past tense.\r\n\r\nWrite few limitations of the study.\r\n\r\nPlease refer to the article “Vitrectomy for Breakthrough Bleeding in Age Related Macular Degeneration and Polypoidal Choroidal Vasculopathy in a Malaysian Hospital” which was published in JSA Vol. 2 No. 2, 2012. Please rephrase your arguments related to this article and whether you are the first researcher to report such or not. Anyway, this above reference has to be cited.\r\n\r\nPlease rectify the English language in your paper.\r\n\r\nReferences should be listed according to alphabetical order.\r\n\r\nThere are no arrows in Figure 3 as described in the figure legend.\r\n\r\nSome parts of the manuscript are very similar to earlier published literature. Please rephrase these sentences because it may lead to plagiarism and unnecessarily publishing conflicts:\r\n\r\n1. “Polypoidal choroidal vasculopathy (PCV) is a retinal disorder characterized by the presence of aneurismal poly-poidal lesions in the choroidal vasculature ……. to be a variant of neovascular age-related macular degeneration (AMD).” (Introduction, 1st paragraph)\r\n\r\n2. “….PCV was defined by the previously determined characteristics of a branching vascular network (BVN) of wide diameter vessels terminating in multiple areas of aneurysmal swelling that were anterior to the choroidal vessels on stereoscopic examination.” (Materials & Methods, 5th paragraph)\r\n\r\nPlease submit the Declaration Form which has to be signed by all authors and the scanned copy has to be e-mailed to the Medicine & Health address. In case, the authors fail to submit, the article will not be published. Declaration Form is a declaration on the part of authors and we weigh it seriously because of legal aspects which may take place, later on. (click the link to download http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/page/2012/02/Declaration%20Form.docx)\r\n'),
(27924, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\n\r\nAppropriate title\r\nMention o-value in the abstract.\r\nPlease submit an abstract and keywords in Bahasa Malaysia (BM) as the journal policy. Only overseas articles are given the benefit of submitting without BM abstract. Since your manuscript is from an institution within Malaysia, we would request you to send an abstract and keywords in BM.\r\n\r\nIntroduction:\r\n\r\nThe use of TRF in this study is not justified. There are other antioxidant available.\r\n\r\nMaterial & Methods/Case:\r\n\r\nPlease mention the age of the rats\r\nPlease indicate (manufacturer name, city, country) for all reagents, chemicals and devices used.\r\nQuantitative estimation of apoptotic cells is mentioned, but no graphical numerical presentation of the results\r\nI am not sure the reason SOD activity is measured in erythrocytes, not in the brain. Please justify.\r\nWhy setting significance value at p<0.01\r\n\r\nResults:  \r\n\r\nNo graphic or numerical presentation at “quantification of apoptotic cells”. just histology images.\r\n\r\nDiscussion:\r\n\r\nIs there any evidence saying that TRF is a metal –chelating agent?\r\nNAC is used as a parallel example, but it is an antioxidant, not metal-chelating agent.\r\nPlease explain/discuss why SOD is used as a single antioxidant index and why measure it in erythrocytes\r\nPlease mention the limitations of the study.	\r\n\r\nReferences:\r\n\r\nPlease write the references conforming to the journal style. The journal does not accept references with numbers. \r\nReferences in the text should contain the authors’ names followed by the year of publication without punctuation mark. Please refer to recent published articles.\r\nReferences should begin on a new page, be double-spaced and listed according to alphabetical order.\r\n\r\nCritical comments:\r\n\r\nDo not start the sentences with abbreviation/ number.\r\nSpacing need to be corrected.\r\n\r\n\r\n\r\n');
INSERT INTO `conference_decision` (`pnid`, `decision`, `feedback`) VALUES
(29687, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\n\r\nAppropriate. But need to delete ‘A case report’.\r\n\r\nSome keywords need to be revised. Please include keywords from MeSH database.\r\n\r\n\r\nIntroduction:\r\n \r\nAdequate \r\n\r\n\r\nMaterial & Methods/Case:\r\n\r\nGood summary; especially clearly indicated the management (mis) by non-ORL team.\r\n\r\n\r\nResults:  \r\n\r\n-\r\n\r\nDiscussion:\r\n\r\nAdequate \r\n\r\n\r\nCritical comments:\r\n\r\nShould also recommend the care of tracheostomy need to be done by trained health care professionals.\r\n\r\nSome parts of the manuscript are very similar to earlier published literature. Please rephrase these sentences because it may lead to plagiarism and unnecessarily publishing conflicts:\r\n\r\n1. “Multiple risk factors are associated …… tight fitting or oversized cannula, excessive tube motion (mechanical irritation), prolonged placement, and disproportionate excision of anterior tracheal cartilage during the creation of the tracheostomy” (Discussion, 2nd paragraph)\r\n\r\n2. “….at the site of the tracheal-tube cuff, where ischemic injury to the tracheal mucosa can take place when cuff pressure exceeds the perfusion pressure of the capillaries of the tracheal wall. With prolonged ischemia, mucosal ulceration, chondritis, and cartilaginous necrosis may ensue, leading to the formation of granulation tissue. Shearing forces from the tube or the cuff may aggravate injury to the airway (Epstein 2005). Granulation tissue may also occur near the distal tip of the TT. Depending on the positioning of the tube, the tip may rub against either the anterior or the posterior tracheal wall. The latter may occur because standard TTs may be too short especially in obese patients with abundant soft tissue in the anterior neck.” (Discussion, 3rd paragraph)\r\n'),
(29954, 2, 'Review comments:\r\n\r\nCorrect the grammar of the sentence “It is also not reported in cancer….” (Abstract)\r\nCheck the last keywords from MeSH.\r\nCorrect the grammar of the sentence “We described a case of CMV retinitis in a HIV-seronegative patient with dermatomyositis and lung malignancy.” (Introduction, 3rd paragraph)\r\nCorrect the spelling of haematology (British)\r\nAlthough you have conclusion, no subheading was given. Please add it.\r\nReference Downes et al. 2016 is not complete. Please include volume number, issue number, and page reference.\r\n'),
(53295, 2, 'Review comments:\r\n\r\nPlease write the abstract in unstructured form (no subheadings needed). \r\n\r\nPlease note that few sentences in the abstract section begin with numerical (“75.1% (n=189) of the participants…; 54.2% had unused medicines and this was due ……; 70.8% of the participants were not given …..; 70% of them disposed their unused medicines in the household garbage, …..”). Grammatically it is incorrect as you can not begin the sentence with numerical.\r\n\r\nPlease submit an abstract in Bahasa Malaysia.\r\n\r\nPlease double check the keywords from MeSH website as few of the keywords were not found in MeSH.\r\n\r\nPlease refer to the sentences “Pharmaceutical Services, Ministry of Health Brunei (MOH), has implemented the ‘Return Your Medicines’ Programme. Through this programme, patients can return their unused or excess medicines, kept at home, for safe disposal by MOH. Among the factors that contribute to excessive medicines in patients’ storage are (i) change of treatment regimen; (ii) cessation of treatment regimen; (iii) non-compliance to their medications; (iv) patient has passed away or (v) medicines obtained from various sources (Polypharmacy)” (Introduction, 2nd paragraph). These are similar to published sentences under MOH, Malaysia. Please rephrase the sentences even if the references are cited. Citations are required.\r\n\r\nIn the methods, please write all the objectives in running text form i.e. i) To find out the participants’ demography;  ii)To identify the number of participants that have been given advice on medicine disposal by medical/health practitioners;  iii) To explore the participants'' knowledge and attitudes towards the methods of disposing…………………………….\r\n\r\nPlease mention how the questionnaire was validated and if it was adapted from any research.\r\n\r\nIn the reference section please include issue number for each journal cited e.g. Abahussain, E. A., D. E. Ball, and W. C. Matowe. 2006. Practice and opinion towards disposal of unused medication in Kuwait. Med Princ Pract 15(5): 352-7.\r\n\r\nPlease include the date accessed in parentheses at the end for reference Naing et al. 2006.   The date the electronic resource was accessed is important because the resource may be changed before someone looking at your sources checks it.  \r\n\r\nPlease abbreviate the names of the journal according to PubMed style.\r\n'),
(38209, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\nThe title is appropriate. \r\nAll the species names in the abstract should be italicized.  \r\n\r\nIntroduction:\r\nSeveral sentences in Introduction should be rewritten: \r\n-	“Worldwide, plant-derived medicines have been increasing of interest  as sources of agents to fight microbial infections” should be changed to “The interest on botanical medicine as a source of antimicrobial agents have been increasing globally.”\r\n-	“The medicinal properties  of this plant indicate it as a valuable source of medicinal compound” should be changed to “Various studies indicated that this plant is a valuable source of medicinal compounds”\r\n-	“The alcoholic stem extract  of Euphorbia tirucalli…” should be changed to “The alcoholic extract of the stem of E.tirucalli…”\r\nConsidering there are many previous studies, please briefly mentioned what are the potential antimicrobial compounds in E.T and their mechanism of action. \r\nPlease provide the justification of selecting the two dental caries bacteria tested. \r\n\r\n\r\nMaterial & Methods/Case:\r\nPlease describe the atmospheric condition for culturing the bacteria. \r\nDid the authors keep a voucher of the plant specimen in the FRIM?\r\nHow many replicates were done for the experiment?\r\n\r\n\r\nResults:  \r\nFigure 1. The upper and lower error bars for positive control are not of equal length. Please edit. \r\nFigure 2. How did the authors estimate the percentage of inhibition? Using mean or individual values against mean of the positive control? If individually there should be error bars. \r\nFigure 2 was not cited in the text.\r\nPlease submit the figures in JPEG files. Figures ‘pasted’ into Word files are not accepted.\r\n\r\nDiscussion:\r\n\r\nConsidering the low percentage of inhibition, does the TE extract has any values in clinical application?\r\n“The presence of various phytochemicals such as flavanoids, polyphenols and phenols in Euphorbia tirucalli stems extracts” The statement is quite vague. Please explore further what are the specific compounds and their mechanism. \r\nThe discussion is incomplete. Please mention the limitation, novelty and potential application of this study. \r\n\r\nPlease add a subheading for conclusion although the text is present.\r\n'),
(42869, 2, 'Review comments 1:\r\n\r\nTitle & Abstract:\r\n\r\nThe Title is OK\r\n\r\nThe Malay abstract needs some improvement, as per suggestions (highlighted in red)\r\n\r\nABSTRAK\r\nObjektif: Kecelaruan spektrum Autisme (ASD) merupakan satu jenis ketidakseimbangan perkembangan neuro kanak-kanak yang dikaitkan dengan kecacatan kognitif dan bahasa. Penyelidikan sebelum ini mendapati bahawa kanak-kanak yang mempunyai ketidakseimbangan perkembangan meningkatkan tahap tekanan ibubapa. Namun, ibubapa yang mempunyai anak ASD mengalami tahap tekanan yang lebih tinggi berbanding ibubapa yang mempunyai kanak-kanak ketidakseimbangan perkembangan yang lain. \r\n\r\nIntroduction:\r\n\r\nAdequate and relevant\r\n\r\n\r\nMaterial & Methods/Case:\r\n\r\nI have one major concern about the statistical test used for the study. The appropriate statistical tool is Chi-square with Yates''s correction (or probably Fischer-exact test), as many of the columns in the cells in the Table 1,2 and 3 have numerical value less than 5. Even though the statistical outcome is not much different, but I think a proper tool should be used to analyze the data. Example, for Table 2:\r\n\r\n11 (36.67)\r\n16 (53.33)\r\n2 (6.67)\r\n1 (3.33)\r\n\r\n\r\nSecondly, after the revision of the statistic was done, it is worth to see the regression analysis for the relevant independent variables, i.e. (PSIDR) (Scores) and parental scores.\r\n\r\n\r\nResults:  \r\n\r\nPlease revise after new statistical manipulation.\r\n\r\nDiscussion:\r\n\r\nAdequate and relevant\r\n\r\nCritical comments: As above. This research is very good with minor typos but must use proper statistical test. The writing styles enable readers to understand the important details and should be applauded. \r\n\r\n\r\nReview comments 2:\r\n\r\nTitle & Abstract:\r\nTitle: I recommend changing the title to ‘Parenting Stress among Malaysian Parents of Children with Autism Spectrum Disorder’ because the original title is too general. \r\nAbstract: Stress level should be translated to ‘tahap tekanan’ instead of ‘kadar tekanan’ because kadar indicates rate (or a time component). \r\nPlease revise the abstract into an unstructured abstract with no subheadings and the total word count should not exceed 250 words for abstract both in English and Bahasa Malaysia (BM).\r\n\r\nIntroduction:\r\nAdequate, but certain statements need clarification: \r\n1.	“Investigating as many aspects of demographic factors as possible is key so that these parents may tackle the stress despite their children’s disorder” Exhaustive collection of demographic factors is not feasible. Please change the statement to “Identifying critical factors contributing to parenting stress enable the parents to handle ASD in their children better”\r\n2.	“Indeed, the current research helps us to… it would not affect their daily lives.” The meaning of this statement is not clear. Suggest to change to ‘Raising children involved identification of ASD and proper handling of the children with this disorder. The current knowledge on ASD has enabled us to identify and embrace children with this disorder. At the same time, we should pay attention to parenting stress among the parents with ASD children’\r\n\r\nMaterial & Methods/Case:\r\n1.	Did the authors calculate the sample size?\r\n2.	Inclusion and exclusion criteria of the subjects should be mentioned clearly. \r\n3.	Did the authors obtain permission to use PSI?\r\n4.	Did the authors obtain permission from MOH before conducting sample collection at the government hospital? \r\n5.	Please provide ethics approval code.  \r\n6.	What is the justification of grouping the subjects according to scores and analysing them using cross-tabulation? Why not comparing the score directly using Mann-Whitney U-test?\r\n7.	Are the cut-off values of the score arbitrary or based on tertiles?\r\n8.	Write down the developer’s name, city and country for SPSS version 21.0\r\n\r\nResults:  \r\n1.	The mean and age range of the parents should be mentioned in the results.\r\n2.	Why ethnicity is not listed as one of the dependent variables? \r\n3.	Did the authors compare parenting stress between parents with male and female ASD children?\r\n4.	Results: “the scale of Defensive Responding indicates the extent…. including his or her spouse (Richard RA, 2001)”. The authors should not interpret the results in this section. Please move to Discussion.  \r\n5.	Tables should be created using the Table function in MS Word. Please use the “insert table” command in Word to create tables. Do not use tabs to create a table. Each piece of data should appear in its own cell. \r\n\r\nDiscussion:\r\nAdequate. \r\n\r\nMinor comments:\r\nThe citation style throughout the manuscript is not standardized. Please edit. \r\nPlease do not start the sentences with abbreviation. \r\nPlease minimized the use of adverbs and adjectives, such as definitely, extremely etc\r\nThere are some minor grammatically errors. Please amend. \r\n\r\n\r\n\r\n'),
(67403, 2, 'Review comments:\r\n\r\nThe review is acceptable but a schematic diagram/table in necessary for this narrative review. Please include such.'),
(67719, 2, 'Review comments:\r\n\r\n1.	The article is informative and interesting.\r\n2.	More data with regard to Malaysia may be added.\r\n3.	Please add the reference Mei Ciu Chang et al. A study of prevalence of anaemia in adolescent girls and reproductive-age women in Kuala Lumpur. Arch Med Sci 2009; 5, 1: 63-68.\r\n4.	Give a subheading for conclusion.\r\n5.	Please submit manuscript in MS Word files.\r\n'),
(32689, 2, 'Review comments:\r\n\r\n1.	In the title, no need to write case report. Please delete the word ‘Case report’.\r\n2.	In the abstract, please mention what is not known and reported in literature and why you are reporting the case. The end sentences should clearly specify what is the take home message for the clinicians. \r\n3.	The keywords need to be revised. Please include keywords from MeSH database.\r\n4.	Introduction is too short. Please expand the text by adding few more references and few more paragraphs.\r\n5.	In the discussion section, it should be clearly stated what is the important of the case and what needs to be remembered. A proper conclusion with subheading should be added. Please cite references in text and reference list according to journal’s style. \r\n6.	Please cite references in text and reference list according to journal’s style.\r\n7.	Please incorporate an old reference “Anxiety and Depression among Patients Before and After\r\nPercutaneous Coronary Intervention (PCI) at National Heart Institute (NHI)” by Ho et al. Med & Health 2007; 2(1): 26-33.\r\n8.	Please add references after 2014. At present, we do not have any reference from 2015 and 2016.\r\n'),
(46084, 3, 'Review comments:\r\n\r\nTitle & Abstract:\r\n\r\nThe title is not suitable: there is no results showing the function of FUM1 gene in this manuscript.\r\nScientific names should be italicized: Fusarium verticillioides.\r\nThe abstract should be in a single paragraph, and no abbreviations should be used.\r\nThis section should be rewritten to summarise the manuscript better, especially in the methodology and result part.\r\nThe authors have not mentioned the treatment of Cuminum cyminum essential oil to the isolates.\r\nIn addition, a conclusion is required for the abstract.\r\n\r\nCorrections:\r\n\r\n- Fusarium is one of the most important pathogens  Fusarium verticillioides is one…\r\n- soil nitrogen contentincrease  increased in soil nitrogen content\r\n- 3 Fusarium non verticillioides samples (negative control)  3 non Fusarium verticillioides samples\r\n- For testing MIC and MFC, the RPMI1640 culture in microplate was used  The antifungal effect of Cuminum cyminum essential oil was determined by broth microdilution testing.\r\n- the cultured RNA samples using the RNA extraction kit of Fermentascompany (Purification Mini Kit GeneJET Plant RNA) was extracted from fungal biomass  RNA was extracted from the fungal biomass\r\n- RT-PCR test along with the FUM1 gene was performed  RT-PCR test for the detection of FUM1 gene expression was performed\r\n- FUM1 gene was expressed in fungal samples that were not under the effects of oil essence  FUM1 gene was expressed in all fungal samples\r\n- gene was not expressed in samples after the cumin impact and there was not any band in agarose gel  gene was not expressed following Cuminum cyminum essential oil treatment\r\n\r\nIntroduction:\r\n\r\nThis section needs major re-arrangement of text.\r\nMajor issues:\r\nThe authors should focus on the background to the research and its aims: i.e. Fusarium verticillioides, Cuminum cyminum essential oil and fumonisin. Infact, the authors have burden the introduction section with categorization of fungus, environmental conditions promoting fungal growth, history of micotoxicology and the methodology of analyzing gene expression.\r\n\r\nCorrections:\r\n\r\n- consumption of contaminated food of fungal  consumption of fungal-contaminated food\r\n- so that the consumer countries have put food safety in top priority and the technical aspects of this issue have overcome its economic aspects  the authors need to rephrase this sentence.\r\n- micotoxin pollution categories have been added to the indicators of the quality of the food  micotoxin pollution have been included as an indicator of food quality.\r\n- Fungi is categorized in the new classification apart from plants and animals  Fungi is categorized in a different kingdom from plants and animals\r\n- To classify fungal food spoilage their appearance would also considered  the authors need to rephrase this sentence.\r\n- The fungus colonized different parts of the plant and it often asymptomatic lesions.  The fungus colonized in different parts of the plant and often present as asymptomatic lesions.\r\n- due to weather conditions, physiological conditions and the amount of nitrogen  due to changes in weather and physiological conditions, and increased amount of nitrogen in soil.\r\n- Important characteristics such as pigment  Important characteristic features of Fusarium species are presence of pigment\r\n- 25 °-C  25°C\r\n- Although a variety of Fusarium are capable of producing fumonisin, but most of them are unknown, and they differ in terms of toxin production.  the authors needs to clarify this statement, and are required to explain further on what is fumonisin.\r\n- In the explanation of Cumin (Cuminum cyminum), the authors have explained the characteristic of its roots and leaves twice: i.e. ‘its root is lanky and white’ and ‘Its root is spindly, more or less branched with 10-20 cm long, lanky and white’; ‘its leaves are thin and stringy with green color’ and ‘Its leaves are alternate, transparent, hairless, leading to very thin incisions and delicate, but it is long and stringy. The leaves are dark green color and its base attached to the stem by a more or less flat sheath’.\r\n\r\nMaterial & Methods/Case:\r\n\r\nAuthors should describe the experimental methods in enough detail that other researchers can replicate results and evaluate claims.\r\n\r\nMajor issues:\r\n\r\n- In this part, the authors did not include the methodology on how the Cuminum cyminum essential oil is produced (or maybe purchased), how the treatment of Cuminum cyminum essential oil was performed in the experiment, fungi RNA isolation, and the RT-PCR protocol.\r\n- The authors have included the methodology of DNA extraction, what is the rationale of performing DNA extraction when the authors have extracted RNA and performed RT-PCR to obtain cDNA?\r\n- The microscopic analysis of fungi was also not mentioned in this section although figures were presented in the result section.\r\n\r\nCorrections:\r\n\r\n- the plates were incubated for 48 h at 35 °  35°C\r\n- scores of 2 (50 percent), scores of 3 (75 percent)  50%, 75%\r\n- The PCR experiments was conducted on extracted RNA to assess DNA contamination  experiments were\r\n- The PCR experiments was conducted on extracted RNA to assess DNA contamination  the authors have mentioned the use of DNAse to remove contaminated DNA, and not by PCR.\r\n- DNase with amount of lμ5  5μl\r\n- The test is performed in a volume of 25-ml  It is impossible for PCR to have a 25 ml reaction\r\n- The primer sequences should be place under 1 table.\r\n\r\nResults:\r\n\r\nMajor issues:\r\n- What is F4 - F18? Are these refers to the 9 different isolates of Fusarium verticillioides? Why is it not named according to F1 - F9? I am guessing the FR200 and FR201 are the standard isolates. Why were the 3 non Fusarium verticillioides samples not included?\r\n- The graph on MIC and MFC: is this a graphical representation of the table? If yes, then it is not required since the authors have presented in table form. In addition, what is the error bars refers to? Is it SEM or SD? This is not explained in the text nor in the figure legend.\r\n- Both microscopic figures show different background colour, why? In addition, the figure for normal fungi was not sharp to appreciate the fungi morphology.\r\n- The author have presented agarose gel picture of VERTF to differentiate the Fusarium verticillioides from the non- Fusarium verticillioides isolates: isolates 3, 4 and 6 still possess a faint 400bp-band. In addition, there are also presence of additional bands (larger than 400bp) in isolate 2, 3, 4 6 and 7.\r\n- The title of this manuscript is on the function of FUM1 gene, although the authors have included the FUM1 gene primers in the previous section, no results have been shown.\r\n\r\nCorrections:\r\n\r\n- 35 degrees Celsius  35°C\r\n\r\nDiscussion:\r\n\r\nThis section needs major improvement to discuss about the results obtained.\r\n\r\nCorrections:\r\n\r\n- spices of Fusarium  species\r\n\r\nCritical comments:\r\nThe English, formulation of certain sentences, and paragraphs need major improvement to provide readers better understandability of the manuscript.\r\nThe authors did not follow the Journal format .\r\nReferencing is not standardized in the manuscript.'),
(54366, 2, 'The reviewer agrees with the scientific merits of the study but the manuscript requires significant rewrite before it can be accepted. \r\n\r\nAbstract: (Malay): Please edit the objective to "kajian ini dilakukan untuk menentukan kesan sindrom metabolic aruhan air minuman fruktos ke atas ekspresi dan aktiviti dan enzim 11β-HSD1 di dalam hati tikus" \r\n\r\nAbstract: It was mentioned 12 rats were used in the Abstract but 18 rats were used in the Methods. Please check the numbers. \r\n\r\nIntroduction: Metabolic syndrome and Cushing''s Syndrome are two similar but unrelated conditions. I think the comparison is unnecessary here. ...The activity of 11β-HSD in cell and tissue was first ''reported''... The authors should acknowledge the original discoverers of 11β-HSD by citing the original papers, not reviews. The authors should elaborate more on the role of 11β-HSD in metabolic syndrome, the research gaps in the field, and how they intended to bridge the gaps using this study. \r\n\r\nMethods: "Fresh fructose drinking water (FDW) was prepared every alternate day"... please edit. Was a particular region of the liver sampled since the enzyme was preferentially expressed near the blood vessels? Was the scoring performed by an anatomist or pathologist? Please mention the manufacturer/developer, city, country of the antibodies, ELISA kit, ELISA reader, SPSS software used. The manufacturer will usually mention the chemicals in each reagents. Please mention those chemicals instead of the "Reagent A/B" or "Substrate Solution" Lower case letter for "mean" Multiple pair-wise comparisons, not multiple group comparisons. \r\n\r\nResults: Please mention the p-value. \r\n\r\nDiscussion: Paragraph 1 and 2 have been covered in the Introduction. They can be removed from the Discussion. The authors should compare and contrast their findings with previous studies, even studies using a different model of MS. Delete "to utter surprise" since the findings are in line with the authors'' hypothesis. The two paragraphs on how fructose drinking water can cause MS should be removed since parameters of MS were not measured in this study. Please add the novelty, clinical importance and limitations of this study in the Discussions.\r\n\r\nConclusion: Please remove limitations from the conclusion paragraph. Figure 2: Please use a simple 2D bar instead of 3D bar.\r\n\r\nReferences: References at the end of the manuscript should be listed according to alphabetical order.\r\n'),
(51980, 2, 'Review comments:\r\n\r\nTiltle : ok\r\nPlease provide a complete affiliation for Dr Aruku Naidu; including the department name, the district address, city, state or country, and postal code.\r\n\r\nAbstract: Malay version, needs to improve.\r\n\r\nThe keywords ''intravascular papillary endothelial hyperplasianeed'' need to be revised as it was not found in MeSH. Please include keywords from MeSH database. \r\n\r\nIntroduction: Acceptable\r\n\r\nCase presentation: Acceptable\r\n\r\nDiscussion:Relevant\r\n'),
(65387, 2, 'Review comments:\r\n\r\nTitle & Abstract:\r\nPATHOGENS FROM CORNEAL SCRAPE SHOW THEIR TRUE COLOURS BETTER IN BACTEC BOTTLES?\r\nSuggest to amend to: DO PATHOGENS FROM CORNEAL SCRAPING SHOW THEIR TRUE COLOURS BETTER IN BACTEC BOTTLES?\r\n  \r\nLatest technique:\r\n\r\nThus we decided to improve culture positive rate for microbial keratitis by adding a new technique using  BD BACTEC Peds Plus/F broth.(include the manufacturer / origin of product including the country)\r\nThe base and leading edges of the ulcer was scraped from the periphery to the center. (this is confusing – the base is usually at the centre)\r\n\r\n\r\nThe scraped material  was smeared on two glass slides, one for potassium hydroxide (KOH) wet mount preparation and the other for Gramstain.\r\n\r\nAll 3 patients eventually recovered without any serious complication such as corneal perforation.\r\n\r\nKartz and associates found this method yields 73.33% positive cultures combined with conventional method together.\r\n\r\nTable 1 was not cited in the text.\r\n\r\nCritical comments:\r\nPlease check your spelling and grammar\r\nPlease include all manufacturer details\r\n\r\n\r\n'),
(7284, 2, 'Review comments:\r\n\r\nPlease begin the abstract with background. Once the background with respect to literature is stated, then the case may be described.\r\nAdd few lines on the clinical features seen in this patient, in the abstract section.\r\nIn the introduction section, there are no references. Please review the literature related to the particular case and cite them with adequate references.\r\nIn the discussion section, please use symbol for percentage (2nd line, 1st paragraph)\r\nThe last reference dates back to 2015. Please add any reference after 2015 and to date.\r\n'),
(42039, 2, 'Review comments:\r\n\r\nPlease rectify the language in the abstract section.\r\n\r\nSentences should be written appropriately. e.g. “We are highlighting…..” should be written as “We highlight……”\r\n\r\nRectify the spelling of pathologists in the abstract section.\r\n\r\nThe manuscript was not written according to the instruction to authors. Following introduction section, the case report section should be there. In the Introduction section, briefly describe what is known in the literature and cite references. Then, in the last paragraph of the Introduction section, mention why you are reporting this case and what is its clinical significance. \r\n\r\nThen, case report section should be there. In this section, mention all clinical features.\r\n\r\nIn the discussion section, please write clearly what is the take home message for a clinician. What needs to be kept in mind with regard to this case should be clearly described or else there is no clinical implication.\r\n\r\nPlease label the finding in the photomicrograph. You may use arrows or other symbols.\r\n\r\nIncorporate latest references from 2016.\r\n');

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(346, 343, '<p>\r\n	Ketebalan kornea memainkan peranan penting dalam merawat pesakit glaukoma. Tujuan kajian in adalah untuk mengenalpasti hubungan di antara ketebalan tengah kornea dengan ukuran ketebalan lapisan saraf retina bagi pesakit glaukoma sudut terbuka (glaukoma bertekanan rendah dan glaukoma bertekanan tinggi). Ini adalah kajian pemerhatian dibuat ke atas pesakit di Pusat Perubatan Universiti Kebangsaan Malaysia dan melibatkan seramai 190 mata (190 pesakit). Pesakit adalah terdiri daripada 60 orang pesakit glaukoma bertekanan rendah, 61 pesakit glaukoma bertekanan tinggi dan seramai 69 pesakit normal yang digunakan sebagai kontrol. Pesakit-pesakit ini dikenalpasti berdasarkan kerosakan luas pandangan yang bercirikan glaukoma dan melalui catatan rekod tekanan mata sebelum rawatan dimulakan. Ketajaman penglihatan diuji. Ketebalan pusat kornea diukur menggunakan mikroskop spekular.Ukuran ketebalan lapisan saraf retina dibuat menggunakan &lsquo;optical coherence tomography. &rsquo;Keputusan kajian menunjukkan pesakit glaukoma bertekanan rendah didapati memiliki perbezaan ketebalan pusat kornea (503.07um&plusmn;32.27) yang ketara, berbanding dengan kumpulan control, 517.45&plusmn;31.74 (p=0.012). Manakala perbezaan ketebalan kornea adalah tidak ketara bagi pesakit glaucoma bertekanan rendah dibandingkan dengan pesakit glaukoma bertekanan tinggi.(p=0.386). Ukuran ketebalan lapisan saraf retina dan pengukuran urat mata menunjukkan perbezaan yang ketara diantara pesakit glaukoma susut terbuka dan kumpulan kontrol. Terdapat korelasi yang ketara di antara ketebalan purata lapisan saraf retina dan ketebalan saraf di bahagian atas dengan ketebalan pusat kornea bagi pesakit glaukoma bertekanan tinggi (r=0.251,0.401) tetapi tidak bagi pesakit glaukoma bertekanan rendah. Kesimpulan kajian ini menunjukkan korelasi ketara bagi ketebalan pusat kornea dengan purata ketebalan lapisan saraf retina dan ketebalan saraf di bahagian atas (&lsquo;superior quadrant&rsquo;) bagi pesakit glaukoma bertekanan tinggi. Korelasi yang serupa tidak ditemui bagi pesakit glaukoma bertekanan rendah.</p>\r\n', 2),
(353, 350, '<p>\r\n	Karsinoma&nbsp; tiub&nbsp; falopian&nbsp; primer&nbsp; jarang&nbsp; ditemui&nbsp; dan&nbsp; sukar&nbsp; didiagnos&nbsp; prabedah.&nbsp; Kami melaporkan satu kes karsinoma tiub falopian primer yang ditemui pada seorang wanita berumur&nbsp; 50&nbsp; tahun&nbsp; yang&nbsp; mempertunjukkan&nbsp; simptom&nbsp; penyakit&nbsp; inflamatori&nbsp; pelvis. Pemeriksaan&nbsp; ultrasound&nbsp; menunjukkan&nbsp; hidrosalfinks&nbsp; bilateral.&nbsp; Walaubagaimanapun, imbasan&nbsp; CT&nbsp; menunjukkan&nbsp; ciri-ciri&nbsp; yang&nbsp; menandakan&nbsp; kanser&nbsp; ovari.&nbsp; Pembedahan dilakukan di mana terdapat dilatasi teruk pada tiub falopian kiri. Pemeriksaan histologi mengesahkan adenokarsinoma serus primer pada tiub tersebut.&nbsp; Bahagian rahim dan kedua-dua&nbsp; ovari&nbsp; adalah&nbsp; bebas&nbsp; dari&nbsp; kanser.&nbsp; Laporan&nbsp; ini&nbsp; menekankan&nbsp; bahawa&nbsp; karsinoma&nbsp; tiub&nbsp; falopian&nbsp; primer&nbsp; harus&nbsp; difikirkan&nbsp; sebagai&nbsp; salah&nbsp; satu&nbsp; diagnosis&nbsp; yang mungkin pada pesakit yang mempertunjukkan simptom penyakit inflamatori pelvis.</p>\r\n', 1),
(360, 357, '<p>\r\n	Sel-sel&nbsp; stem&nbsp; leukemia&nbsp; mempunyai&nbsp; pelbagai&nbsp; potensi&nbsp; diferensiasi.&nbsp; Imunofenotip&nbsp; sel leukemia&nbsp; biasanya&nbsp; konsisten&nbsp; sepanjang&nbsp; riwayat&nbsp; penyakit&nbsp; walaupun&nbsp; pada&nbsp; peristiwa kambuh.&nbsp; Jarang-jarang&nbsp; sekali,&nbsp; sel&nbsp; leukemia&nbsp; mungkin&nbsp; mengalami&nbsp; &#39;lineage&nbsp; switch&#39; terutamanya&nbsp; pada&nbsp; saat&nbsp; kambuh.&nbsp; Kami&nbsp; ingin&nbsp; menyoroti&nbsp; kes&nbsp; tersebut&nbsp; pada&nbsp; seorang kanak-kanak lelaki berusia 10 tahun yang mengalami kelesuan, hilang selera makan, demam,&nbsp; gangguan pernafasan,&nbsp; kegagalan&nbsp; jantung,&nbsp; edema&nbsp; dan hepatosplenomegali selama&nbsp; dua minggu.&nbsp; Keputusan&nbsp; darah&nbsp; menunjukkan&nbsp; jumlah&nbsp; leukosit&nbsp; 41.5x109/l&nbsp; dan platelet&nbsp; 37x109/l.&nbsp; Filem&nbsp; darah&nbsp; periferi&nbsp; dan&nbsp; sampel&nbsp; aspirasi&nbsp; sumsum&nbsp; tulang menunjukkan&nbsp; kehadiran&nbsp; sel&nbsp; leukemia&nbsp; dalam&nbsp; kuantiti&nbsp; yang&nbsp; banyak.&nbsp; Sel-sel&nbsp; itu mempunyai&nbsp; nisbah&nbsp; nukleus&nbsp; kepada&nbsp; sitoplasma&nbsp; yang tinggi.&nbsp; Imunofenotip&nbsp; dan sitokemia&nbsp; menunjukkan&nbsp; keputusan&nbsp; yang&nbsp; konsisten&nbsp; dengan&nbsp; diagnosis&nbsp; T-sel&nbsp; leukemia limfoblastik akut. Pesakit mencapai remisi selepas dirawat dengan kemoterapi rejim B &lsquo;UK&nbsp; ALL 97&nbsp; protocol&rsquo;.&nbsp; Namun,&nbsp; penyakitnya&nbsp; kambuh&nbsp; tujuh&nbsp; bulan&nbsp; selepas&nbsp; itu&nbsp; dengan kehadiran&nbsp; sel&nbsp; leukemia&nbsp; sebanyak&nbsp; 26%&nbsp; di&nbsp; dalam&nbsp; sampel&nbsp; aspirat&nbsp; sumsum&nbsp; tulang. Majoriti adalah sel leukemia jenis L1 bercampur dengan minoriti sel leukemia jenis L2.&nbsp; Imunofenotip&nbsp; konsisten&nbsp; dengan&nbsp; leukemia&nbsp; prekursor&nbsp; B limfoblastik&nbsp; akut.&nbsp; Rawatan ditukar&nbsp; kepada&nbsp; kemoterapi&nbsp; yang&nbsp; lebih&nbsp; khusus,&nbsp; namun,&nbsp; pesakit&nbsp; tidak mencapai&nbsp; remisi dan&nbsp; kemudiannya&nbsp; rawatannya&nbsp; ditukar&nbsp; kepada&nbsp; kemoterapi&nbsp; paliatif.&nbsp; Kes&nbsp; ini mengilustrasikan&nbsp; kes&nbsp; &#39;lineage&nbsp; switch&#39;&nbsp; awal&nbsp; dari&nbsp; T-sel&nbsp; limfoblastik&nbsp; leukemia&nbsp; akut kepada leukemia prekursor B limfoblastik akut.</p>\r\n', 1),
(368, 365, '<p>\r\n	Ini adalah satu kajian retrospektif untuk melihat presentasi kanser payudara dan cara ianya dikesan dikalangan pesakit di Hospital Kuala Lumpur. Sebanyak 366 rekod pesakit dari Klinik Endokrin dan Payudara dianalisa. Hasil kajian menunjukan kebanyakan pesakit adalah dalam lingkungan umur 40-49 tahun (39.6%). Sebanyak 81.4% pesakit menunjukkan ketulan pada payudara dan mengesan ketulan ini sendiri (97.3%). Diameter min tumor semasa presentasi adalah 4.7&plusmn;3 cm.Sebanyak 1.6% penyakit di kesan oleh kakitangan perubatan dan 1.1% lagi dikesan melalui mamogram. Kesimpulannya, kebanyakan pesakit mengesan ketulan pada payudara mereka sendiri. Ini mencadangkan bahawa kaedah pemeriksaan sendiri payudara boleh digunakan sebagai saringan untuk mengesan tanda-tanda awal kanser payudara, sekiranya kemudahan mamogram tidak ada. Pengesanan awal melalui pemeriksaan sendiri dapat menawarkan pilihan rawatan dan kualiti hidup yang lebih baik sungguhpun terdapat bukti yang menyatakan bahawa kaedah ini tidak dapat mengurangkan mortaliti kanser payudara.</p>\r\n', 1),
(369, 366, '<p>\r\n	Translokasi salingan t(9; 22)(q34; q11) yang&nbsp; menghasilkan kromosom Philadelphia (Ph<sup>1</sup>) dan juga gen lakuran BCR/ABL memainkan peranan utama dalam diagnosis dan patogenesis penyakit leukemia myeloid kronik (CML). Didalam kajian ini, kami mengkaji peranan teknik&nbsp; pendaflor <em>in situ</em> hibridasasi (FISH) dalam mengesan gen lakuran BCR/ABL pada 18 sampel aspirasi sumsum tulang daripada pesakit CML dan pesakit yang disyaki menghidap CML. Kami juga membuat analisa perbandingan mengenai kesensitifan, spesifisiti dan kadar pengesanan gen lakuran BCR/ABL di antara teknik penghibridan pendaflor (FISH), kariotip sitogenetik dan tindak balas berantai polimerase (PCR).Sensitiviti, spesifisiti dan kadar pengesanan untuk FISH adalah 100% untuk kesemuanya. Untuk kariotip sitogenetik pula, sensitiviti, spesifisiti dan kadar pengesanan adalah 85%, 100% dan 100% masing- masing. Kariotip sitogenetik juga telah mengesan sejenis aberasi kromosom di samping Ph<sup>1</sup>. Kajian ini menunjukkan kaedah FISH adalah sangat sensitif dalam mengesan gen lakuran BCR/ABL. Kariotip sitogenetik tetap menjadi salah satu kaedah utama dalam penyiasatan pesakit CML kerana terdapat kebarangkalian mengesan aberasi kromosom disamping Ph<sup>1</sup>. Oleh itu, kariotip sitogenetik dan FISH adalah dua teknik (komplementari) yang saling melengkapi dalam pengesanan gen lakuran BCR/ABL pada pesakit CML.</p>\r\n', 1),
(370, 367, '<p>\r\n	Jagarawatan berkualiti akan memberi kesan mendalam terhadap kepuasan pesakit. Jagarawatan berkualiti dianggap oleh pesakit pos kardiak sebagai keperluan dari segi fizikal, emosi dan rohani yang perlu di penuhi ketika berada di hospital. Objektif kajian ini adalah untuk mengenalpasti faktor-faktor jagarawatan berkualiti yang mempengaruhi tahap kepuasan pesakit pos kardiak. Kajian deskriptif keratan rentas ini menggunakan instrumen &lsquo;NURSQUAL&rsquo; yang merangkumi empat elemen iaitu kecekapan teknikal (technical competence), pemberian maklumat (information giving), jaminan (assurance) dan empati (empathy), untuk menilai tahap kepuasan pesakit terhadap jagarawatan berkualiti. Ianya merangkumi empat soalan kajian dengan 33 item untuk mengukur keempat-empat elemen tersebut. Daripada Januari hingga Mac 2005, seramai 52 pesakit pos kardiak yang telah menjalani pembedahan jantung di IJN yang memenuhi kriteria telah mengambil bahagian dalam kajian ini. Hasil kajian menunjukkan perbezaan yang signifikan diantara elemen kecekapan teknikal (technical competence), pemberian maklumat (information giving), jaminan (assurance) dan empati (empathy) dengan kepuasan pesakit dengan nilai p &lt; 0.05. Elemen jaminan (assurance) memberi sumbangan statistik yang kukuh dan unik dalam menentukan kepuasan pesakit dengan nilai p &lt; 0.05.</p>\r\n', 1),
(371, 368, '<p>\r\n	Kajian ke atas 50 pesakit <em>Non-Insulin Dependent Diabetes Mellitus </em>(NIDDM) yang &nbsp;menjalani pembedahan di bawah anestesia (pelalian) epidural telah dijalankan. Kesemua pesakit diberi infusi dekstros 5% 100ml/jam dari masa berpuasa hingga pesakit tiba di bilik bedah. Pesakit dibahagikan kepada dua kumpulan secara rawak. Pesakit dalam Kumpulan 1 (n=25) menerima salin normal manakala pesakit Kumpulan 2 (n=25) diberi laktat Ringer. Kedua-dua kumpulan menerima infusi sepanjang masa pembedahan sehingga empat jam selepas pembedahan. Aras glukos darah diambil tiga kali, iaitu semasa permulaan (baseline), 45 minit semasa pembedahan sedang berjalan, 30 minit dan empat jam selepas pembedahan menggunakan glukometer. Hasil kajian menunjukkan paras glukos darah min 1.5 mmol/L lebih tinggi pada pesakit Kumpulan 2 di antara empat jam selepas pembedahan dan permulaan, (baseline) berbanding dengan 0.96 mmol/L di kalangan pesakit Kumpulan 1. Walau bagaimanapun, statistik menunjukkan keputusan ini adalah tidak signifikan. Tidak terdapat perbezaan paras glukos min pada 30 minit bila dibezakan dengan permulaan (baseline). Paras glukos darah min menunjukkan kenaikan yang signifikan pada kedua-dua kumpulan selepas pembedahan apabila dibezakan dengan paras glukos di waktu permulaan. Hasil kajian ini menunjukkan bahawa pesakit NIDDM yang menerima laktat Ringer mempunyai kenaikan paras glukos darah min yang lebih besar apabila dibezakan dengan pesakit yang menerima salin normal, akan tetapi, magnitudnya tidak signifikan dari segi statistik.</p>\r\n', 1),
(372, 369, '<p>\r\n	Sebahagian dari pesakit yang mengalami deman akut dengan trombositopenia, tidak semestinya mengidap penyakit denggi. Merawat mereka seperti mana mereka mengidap denggi adalah tidak wajar. Prevalens penyakit tersebut masih tidak diketahui. Objektif kajian ini adalah untuk menentukan prevalens penyakit deman akut dengan trombositopenia yang&nbsp; bukan denggi di kalangan pesakit deman akut tidak spesifik dari klinik pesakit luar. Ini adalah satu kajian keratan rentas di klinik. Semua pesakit yang mengadu deman kurang dari dua minggu yang tidak spesifik dari Pusat Perubatan Primer Hospital Universiti Kebangsaan Malaysia (HUKM) dan Klinik kesihatan Batu 9 Hulu Langat dipilih untuk mengambil bahagian. Kajian kiraan darah lengkap (full blood count) pada hari pertama mereka ke klinik dan serologi denggi pada hari ke 5 selepas demam bermula dijalankan. Seramai 73 pesakit berjaya mengambil bahagian dari Mei hingga November 2003. Di antara mereka, 47.9% (35) pesakit didapati mempunyai trombositopenia. Seramai 40% (14) dari mereka yang mempunyai trombositopenia adalah negatif dalam ujian serologi denggi. Maka, pervalens penyakit deman akut dengan trombositopenia yang&nbsp; bukan denggi adalah 19.2%. Dengan ketaranya, sehagian pesakit di kalangan pesakit deman akut tidak spesifik, menunjukkan ujian serologi denggi negatif. Pesakit-pesakit ini harus dibezakan dari kalangan pesakit deman akut tidak spesifik, kerana mereka mungkin berbeza dari segi riwayat penyakit dan rawatanya.</p>\r\n', 1),
(373, 370, '<p>\r\n	Diagnosis kanser payu dara di kalangan wanita Malaysia telah mendorong ramai wanita untuk mendapatkan rawatan daripada pelbagai pertubuhan kesihatan dan ianya merupakan satu amalan yang ketara di kalangan wanita Malaysia. Objektif kajian ini adalah untuk mengetahui sama ada penyembuhan adalah satu maklumat yang penting kepada wanita yang baru didiagnos dengan penyakit kanser payudara. Kajian ini juga, bertujuan untuk menilai sama ada maklumat mengenai penyembuhan dipengaruhi oleh variabel lain seperti kumpulan etnik, umur, tahap pendidikan dan kelas sosial. Kaedah yang digunakan dalam kajian ini adalah kaedah temubual yang berstruktur dengan sembilan item maklumat, diberikan kepada 100 orang wanita yang baru didiagnos dengan kanser payudara. Wanita-wanita ini juga diminta memilih maklumat penting yang menjadi keutamaan. Kaedah Thurstone Scaling digunakan untuk menghasilkan profil maklumat mengikut pilihan dan skala nilai mengikut keutamaan. Analisis Varians (ANOVA) dan ujian-t telah digunakan untuk melihat perbezaan antara skor min. Keputusan kajian mendapati bahawa maklumat tentang kecenderungan ke arah penyembuhan mendapat keutamaan di kalangan responden. Hasil kajian juga mendapati tiada perbezaan yang signifikan di antara tahap pendidikan, kelas sosial, umur, serta kumpulan etnik dengan maklumat tentang penyembuhan. Kesimpulan kajian ini menjelaskan bahawa maklumat penyembuhan penting bagi wanita-wanita yang baru didiagnos dengan kanser payudara tanpa mengira kumpulan etnik, umur, tahap pendidikan dan kelas sosial. Hasil kajian ini dapat membantu para doktor dan jururawat memberi maklumat dan nasihat kepada wanita yang baru didiagnos dengan kanser payudara.</p>\r\n', 1),
(374, 371, '<p>\r\n	Sindrom &lsquo;Fragile X&rsquo; lazimnya disebabkan oleh ketidakstabilan kembangan jujukan ulangan trinukleotid pada gen FMR-1. Warisannya adalah rantaian X diturunkan secara &lsquo;reduce penetrance&rsquo; dan frekuensinya terus meningkat melalui beberapa generasi. Fenotip yang dimanifestasikan oleh sindrom &lsquo;fragile X&rsquo; adalah tidak spesifik. Penganalisaan secara sitogenetik memerlukan pengalaman untuk mengenalpasti tapak rapuhan pada metafasis manakala analisis DNA seperti Pemblotan &lsquo;Southern&rsquo; adalah sangat mahal apabila ia digunakan sebagai rujukan rutin saringan. Kajian ini bertujuan untuk memperkenalkan teknik berasaskan tindakbalas rantaian polimerase (&lsquo;Touch down PCR&rsquo;) sebagai kaedah saringan sindrom &lsquo;Fragile X&rsquo;. Sejumlah enam kes telah dikajii. Antaranya, satu kes merupakan kes sindrom Fragile X (T1) yang telah dikenalpasti dengan kaedah sitogenetik konvensional, dua lagi merupakan ibu (T2) dan bapanya (T3). Dua kes yang lain (T4 dan T5) telah dipilih secara rawak daripada pesakit yang masing-masing menunjukkan ciri-ciri dismorfik dan kerencatan perkembangan. Satu kawalan normal (TC) juga dimasukkan. Analisis sitogenetik terhadap tapak rapuhan telah dijalankan. Dua sistem kultur digunakan, termasuk kultur &lsquo;synchronized&rsquo; limfosit dan kultur kurangan folat-thimidin. Pencelupan metafasa dari kultur &lsquo;fragile X&rsquo; dianalisis untuk kehadiran tapak rapuhan pada kromosom X. Kariotip daripada pencelupan saluran-G dibuat dengan alat penganalisa imejan untuk kesemua sampel yang dikaji bagi menyingkirkan kehadiran ketidaknormalan kromosom. DNA diekstrak daripada sampel-sampel ini dan digandakan dengan rantai polimerase &lsquo;Touch-Down&rsquo;. Analisis sitogenetik menunjukkan satu tapak rapuhan sensitif-folat pada lelaki yang disyaki dan tapak rapuhan negatif pada lima sampel yang lain. Kariotip daripada pencelupan saluran-G menunjukkan tiada tambahan ketidaknormalan kromosom. Kesemua DNA yang diekstrak mengganda dengan baik. Lima daripada sampel kajian tapak rapuhan negatif menunjukkan kehadiran produk pada saluran yang dijangka iaitu 522bp, yang menunjukkan tiada kembangan gen FRM-1. Namun, tiada produk PCR dikesan pada lelaki yang dijangkiti; keputusan ujian PCR menunjukkan beliau mungkin mengalami kembangan penuh gen FMR-1. Kajian kami menunjukkan bahawa ujian PCR adalah satu kaedah yang cepat dan&nbsp; spesifik untuk menyaring alel normal dan mutasi penuh pada gen &lsquo;Fragile X&rsquo;. Kami mencadangkan ia digunakan sebagai kaedah pelengkap kepada analisis sitogenetik untuk mengesan gen FRAXA (<em>Folate Sensitive Fragile Site at Xq28</em>).</p>\r\n', 1),
(375, 372, '<p>\r\n	Kajian ini bertujuan untuk menunjukkan peranan kaedah hibridisasi in situ pendaflor (FISH) dan sitogenetik konvensional (karyotip) pada kes sindrom mikrodelesi yang telah disahkan secara sitogenetik dan klinikal. Sejumlah sembilan kes telah dirujuk dari tahun 2002 hingga 2004 di Unit Sitogenetik, Hospital Universiti Kebangsaan Malaysia (HUKM). Kes-kes tersebut terdiri daripada tiga sindrom Prader-Willi, tiga sindrom DiGeorge, satu sindrom Williams , satu sindrom Miller-Dieker&nbsp; dan satu sindrom Kallmann. Sampel darah kesemua pesakit telah dikultur dan dituai mengikut prosedur standard. Sebanyak 20 serakan metafasa telah dianalisa untuk setiap kes. Analisa FISH dilakukan untuk kesemua kes menggunakan prob komersial (Vysis,USA): SNRPN dan D15S10 untuk sindrom Prader-Willi, LIS1 untuk sindrom Miller-Dieker, ELN untuk sindrom Williams, KAL untuk sindrom Kallmann, TUPLE1 dan D22S75 untuk sindrom DiGeorge. Analisa sitogenetik konvensional menunjukkan kariotip normal untuk kesemua kes kecuali satu yang menunjukkan aberasi kromosom melibatkan kromosom 9 dan 22. Analisa FISH menunjukkan mikrodelesi pada kesemua sembilan kes. Kajian ini menunjukkan dua fakta penting, iaitu hibridisasi in situ pendaflor (FISH) merupakan kaedah yang mesti dilakukan untuk pengecaman sindrom mikrodelesi, manakala kaedah sitogenetik konvensional merupakan kaedah saringan bagi keabnormalan kromosom yang mungkin berkaitan dengan penyakit tersebut.</p>\r\n', 1),
(376, 373, '<p>\r\n	Kami melaporkan dua kes leukemia akut bifenotipik yang didiagnos di Hospital Universiti Kebangsaan Malaysia (HUKM), ciri-ciri klinikal, hematologi dan&nbsp;tindakbalas penyakit ini terhadap kemoterapi. Kedua-dua pesakit adalah wanita pertengahan umur yang menunjukkan pembengkakan hati dan limpa serta bilangan sel darah putih yang tinggi. &nbsp;Sum-sum tulang menunjukkan kepadatan sel dimana 90% terdiri dari sel-sel leukemia pelbagai saiz. Analisa sitokimia menunjukkan lebih daripada 3% adalah positif terhadap ujian peroksidase dan sel-sel leukaemia yang lebih kecil menunjukkan positiviti secara blok terhadap ujian <em>periodic acid-schiff</em> (PAS). &nbsp;Ujian imunofenotip menunjukkan ko-ekspresi dwi penanda sel CD 33 dan CD 19, CD 19 dan CD34, intra CD22, intra TdT dan intraMPO. &nbsp;Analisa sitogenetik daripada salah seorang pesakit tersebut menunjukkan kehadiran kromosom Philadelphia &nbsp;yang disahkan dengan ujian Fluorescence <em>In Situ</em> Hybridisation (FISH). Analisa molekular&nbsp;juga menunjukkan kehadiran protein fusi BCR-ABL. &nbsp;Kedua-dua pesakit menerima rawatan kemoterapi yang terdiri daripada daunorubicin dan cytosine arabinoside. Namun begitu, pesakit yang mempunyai kromosom Philadelphia BCR-ABL tidak berjaya mencapai remisi morfologi selepas kemoterapi induksi. Memandangkan prognosis buruk pesakit leukemia ini, &nbsp;pemindahan sel-sel stem periferi dirancangkan untuk kedua-dua pesakit ini.</p>\r\n', 1),
(377, 374, '<p>\r\n	Pengerasan pada koklea sebelum ini adalah kontra indikasi relatif bagi pembedahan implan koklea. Ini disebabkan oleh kesulitan untuk memasukkan elektrod dengan selamat ke dalam koklea. Akibatnya elektrod atau bahagian telinga dalam berkemungkinan akan rosak. Tambahan pula osifikasi pada skala timpani akan menghadkan bilangan elektrod yang masuk ke dalam koklea.&nbsp; Keberkesanan implan koklea dipertikaikan disebabkan kemungkinan jumlah aliran elektrik yang tinggi diperlukan di dalam koklea yang telah mengalami pengerasan. Di dalam kes ini juga jumlah sel neuron &nbsp;tidak diketahui. Ini semua mungkin akan menyebabkan kesulitan pada prosedur pembedahan dan memberi kesan pada hasil jangka masa panjang.&nbsp; Walaubagimanapun sekarang ini pembedahan implan koklea dapat dilakukan pada koklea yang telah mengalami osifikasi bergantung kepada pengalaman doktor pakar bedah. Disini kami melapurkan bahawa pembedahan implan koklea adalah selamat dijalankan pada koklea yang telah mengalami pengerasan.</p>\r\n', 1),
(378, 375, '<p>\r\n	Perkaitan antara papilomavirus manusia (HPV) dengan papilomatosis larinks telah lama didokumentasi. Kami melaporkan dua kes papilomatosis larinks dan mengkorelasi kes-kes ini dengan kehadiran HPV, protein-protein p53 dan c-erbB-2. Kes yang pertama ialah seorang pesakit lelaki berumur satu tahun dan kes yang kedua, seorang pesakit perempuan berumur enam tahun. Spesimen biopsi yang berfiksasi formalin dan terbenam-parafin telah diuji secara teknik in situ hibridisasi untuk kehadiran genom HPV. Teknik ini menggunakan prob-prob berbiotin yang spektrumnya luas dan jenis spesifik. Manakala ekspresi imunohistokimia pada protein-protein p53 (DO7, 1:50) dan c-erbB-2 (DAKO A0485, 1:300) dievaluasi dengan antibodi yang terdapat secara komersial. Secara histologi, tumor pada kedua-dua kes ini menunjukkan bentuk papilari pada papiloma skuamus. Kes pertama mengesan HPV jenis 6, HPV jenis 11 dan ekspresi protein p53 manakala kes kedua hanya mempunyai HPV jenis 6. Kedua-dua kes HPV menunjukkan tanda positif terhad di nukleus pada epitelium skuamus superfisial. Kes yang pertama menunjukkan kepositifan p53 yang kelihatan di bahagian basal hingga satu per tiga epitelium papiloma larinks dan biopsi susulan yang &nbsp;terkini menunjukkan kepositifan p53 telah melanjut ke lapisan atasan epithelium. Ekspresi protein c-erbB-2 tidak dikesan pada kedua-dua kes ini. Penemuan ini adalah sama dengan kajian lain di mana pemantauan susulan klinikal pada kes-kes ini disyorkan kerana ia kecenderungan berulang.</p>\r\n', 1),
(379, 376, '<p>\r\n	Sitologi sedutan jarum halus di bawah panduan radiologik untuk mendiagnosa karsinoma sel renal telah diterima pakai dan digunakan secara meluas. Ini adalah kerana karsinoma sel renal mempamirkan ciri sitologi yang karakteristik yang membolehkan ianya di identifikasi secara sitologi dengan tepatl. Kami membentangkan kes seorang lelaki berumur 66 tahun yang menghidap karsinoma sel renal peringkat lanjut dimana&nbsp; telah&nbsp; terdapat penyebaran sel kanser ke kelenjar limfa aorta dan servikal, peparu dan hepar. Sitologi sedutan jarum halus dari massa para-aortik menunjukkan gugusan terlekat sel malignan dengan sitoplasma yang banyak dan bervakuol konsisten dengan diagnosis karsinoma sel renal. Histologi dari kelenjar limfa servikal dan dengan penemuan ujian imunohistokimia, diagnosisnya adalah konsisten dengan karsinoma sel renal metastatik. Pesakit meninggal dunia tiga tahun tahun selepas diagnosis dibuat.</p>\r\n', 1),
(380, 377, '<p>\r\n	Mielofibrosis adalah sejenis penyakit mieloproliferatif kronik yang dicirikan dengan pembengkakan limpa, anemia leukoeritroblastik, poikilositosis berbentuk air-mata (tear-drop poikilocytosis) dan fibrosis sumsum tulang. Pembedahan splenektomi dilakukan untuk pesakit yang memerlukan transfusi darah yang kerap ataupun pembengkakan limpa yang amat besar sehingga mengakibatkan gejala-gejala yang memilukan serta trombositopenia yang teruk akibat&nbsp; hipersplenisma.Seorang wanita berumur 56 tahun telah didiagnos menghidap penyakit mielofibrosis primer pada tahun 2000 dan dirujuk ke Hospital UKM untuk rawatan selanjutnya bagi kekerapan transfusi darah yang semakin&nbsp; meningkat dan juga kerana pembengkakan limpanya yang teruk. Beliau juga telah mengalami kolesistitis akut sebanyak dua kali pada masa yang lampau. Pemeriksaan menunjukkan paras hemoglobin 6.4g/dl, kiraan sel darah putih : 23.4x10<sup>9</sup>/l, kiraan platelet : 163x10<sup>9</sup>/l dengan gambaran leukoeritroblastik dan poikilositosis berbentuk air-mata (tear-drop poikilocytosis) pada filem darah periferi. Bacaan alkalin fosfatase neutrofil (Neutrophil alkaline phosphatase NAP score) adalah 184/100 neutrofil. Ujian ultrasound pada abdomen menunjukkan splenomegali yang teruk, dan juga terdapat batu karang pundi hempedu yang multipel. Memandangkan kekerapan transfusi darah yang semakin meningkat dan juga gejala splenomegali yang teruk, pembedahan splenektomi dan kolesistektomi telah dilakukan. Pemeriksaan filem darah periferinya selepas splenektomi menunjukkan penurunan poikilositosis berbentuk air-mata (tear-drop poikilocytosis) dan juga kehilangan gambaran leukoeritroblastosis. Kami percaya bahawa limpa memainkan peranan yang penting dalam pembentukan poikilositosis berbentuk air-mata (tear-drop poikilocytosis) dan leukoeritroblastosis, tetapi mekanisma yang terlibat masih tidak jelas.</p>\r\n', 1),
(381, 378, '<p>\r\n	Adenokarsinoma papilari-viloglandular telah pertama kali dihuraikan oleh Young dan Scully pada tahun 1989 sebagai suatu entiti tersendiri dengan ciri-ciri histologi seperti pertumbuhan eksofitik, struktur vilus dan papilari serta atipia nukleus yang sedikit ke sederhana. Kami melaporkan satu kes adenokarsinoma papilari-viloglandular serviks dengan metastasis ke nodus limfa dan mengkajisemula ciri-ciri klinikopatologi enam kes yang telah dilaporkan. Pesakit ini merupakan kes ketujuh dengan ciri-ciri yang sama seperti kes-kes yang telah dilaporkan. Umur ketujuh-tujuh pesakit adalah diantara 29 hingga 54 tahun (min, 41). Diantara tujuh kes ini, lima mempunyai invasi limfovaskular. Semua kecuali pesakit ketujuh telah dirawat dengan histerektomi radikal dan radioterapi. Jagaan susulan pesakit ketujuh adalah 7 bulan, malangnya beliau &nbsp;telah meninggal dunia akibat infeksi respiratori.</p>\r\n', 1),
(382, 379, '<p>\r\n	Dua laporan kes vaskulitis retina pada tuberkulosis okular presumptif dibentangkan. Kes 1 ialah seorang wanita Melayu berumur 26 tahun yang mengadu flebitis dengan lesi koroid. Kes 2 ialah seorang wanita Melayu berumur 27 tahun yang mengadu sakit arteritis oklusif. Kedua-dua pesakit menunjukkan ujian Mantoux positif dengan saringan sistemik negatif. Kedua-duanya menunjukkan respons baik terhadap terapi anti-tuberkulosis beberapa hari selepas terapi dimulakan. Tetapi serentak dengan kejadian ini, peningkatan vitritis berlaku. Vitritis menunjukkan penurunan selepas sahaja kortikosteroid oral diberi. Cara presentasi tuberkulosis okular kedua-dua kes ini jarang ditemui dan indeks kecurigaan yang tinggi diperlukan. Terapi anti-tuberkulosis pelbagai ubat perlu digabungkan dengan kortikosteroid oral untuk rawatan efektif.</p>\r\n', 1),
(383, 380, '<p>\r\n	Jangkitan daripada virus hepatitis B (HBV) merupakan satu isu kesihatan besar sedunia. HBV merupakan suatu faktor terhadap peningkatan risiko&nbsp; penyakit hepar seperti sirosis dan juga dalam perkembangan penyakit kanser hepatosellular. Masalah ini terutamanya tersebar luas di rantau Asia-Pasifik, termasuk Malaysia. Semasa jangkitan, e antigen hepatitis B (HBeAg) adalah dihasilkan di dalam perumah. Antigen ini adalah satu penanda serologi yang penting di dalam diagnosis kesakitan kronik hepatitis B. Perubahan (seroconversion) kepada anti-bodi (anti-HBe) merupakan satu faktor terhadap kemajuan prognosis pengyakit. Tetapi terdapat beberapa mutasi seperti dwi mutasi&nbsp; ke atas promoter teras (core promoter double mutations) (A<sup>1762</sup>G<sup>1764</sup>&rarr;T<sup>1762</sup>A<sup>1764</sup>), varian kodon 15 (C<sup>1858</sup>/ T<sup>1858</sup>) dan mutasi pada kodon henti ke atas pra-teras (pre-core stop codon mutation) (T<u>G</u>G&rarr;T<u>A</u>G) yang boleh memberi kesan kepada pengeluaran HBeAg. Ini boleh mengakibatkan implikasi terhadap diagnosis dan klinikal penyakit tersebut. Selain itu, HBV juga boleh dibahagikan kepada lapan genotip (A ke H), subjenis genotip dan rekombinan&nbsp; juga ditemui. Kajian telah menunjukkan bahawa genotip&nbsp; berbezaan dari segi hubungan dengan mutasi-mutasi tersebut dan juga dengan prognosis penyakit hepatitis B.&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n', 1),
(384, 381, '<p>\r\n	&ldquo;Percutaneous coronary intervention&rdquo; (PCI) dikenalpasti sebagai salah satu prosedur diagnostik invasif yang menimbulkan kebimbangan yang tinggi di kalanganpesakit jantung. Secara tidak langsung, peningkatan tahap kebimbangan ini akan menyebabkan pesakit merasa tertekan dan lantas, mengalami kemurungan. Objektif kajian ini adalah untuk mengenalpasti&nbsp; tahap kebimbangan dan tahap tekanan pesakit sebelum dan selepas PCI. Kajian deskriptif keratan rentas ini menggunakan instrumen &ldquo;Hospital Anxiety and Depression Scale&rdquo; (HADS) yang mengandungi dua elemen berkaitan dengan tahap kebimbangan dan tahap tekanan pesakit sebelum dan selepas PCI. Kajian ini telah dijalankan di wad kardiologi Anggerik dan Dahlia dan Jagaan Harian di Institut Jantung Negara (IJN) dari Januari hingga Februari 2006. Ia melibatkan seramai 61 responden yang memenuhi kriteria yang telah ditetapkan sebelum dan selepas PCI. Hasil kajian mendapati 40 dan 38 pesakit sebelum dan selepas PCI mengalami tahap kebimbangan yang rendah; seramai 47 dan 46 pesakit sebelum and selepas PCI turut mengalami tahap tekanan yang rendah. Tahap kebimbangan dan tahap tekanan sebelum and selepas PCI tiada perbezaan signifikan dengan faktor sosio demografik responden.&nbsp; Manakala, didapati korelasi yang signifikan (p &lt; 0.05) di antara tahap kebimbangan dan tahap tekanan sebelum dan selepas PCI. Namun begitu, tahap kebimbangan dan tekanan yang rendah di kalangan pesakit sebelum dan selepas PCI di Insititut Jantung Negara (IJN) mungkin dipengaruhi oleh faktor seperti, kemudahan yang lengkap dan terkini serta pendidikan jagaan kesihatan yang diberikan oleh pakar kardiologi, jururawat dan juruteknik kardiak.</p>\r\n', 1),
(385, 382, '<p>\r\n	Kehamilan merupakan satu pengalaman yang mencemaskan, namun membawa kegembiraan bagi hampir semua ibu-ibu yang mengharapkan cahaya mata. Walaupun begitu, bukan semua ibu mengalami kegembiraan. Terdapat juga setengah ibu yang mengalami tahap tekanan dan kebimbangan yang tinggi tanpa sebab diketahui. Tujuan kajian ini dijalankan adalah untuk mengenalpasti tahap tekanan dan kebimbangan dikalangan wanita hamil yang berisiko tinggi dan faktor-faktor yang menyebabkan tekanan dan kebimbangan.&nbsp; Satu kajian rentas deskriptif telah dijalankan keatas 38 wanita hamil yang berisiko tinggi yang telah berada diwad obstetrikselama tiga hari, Hospital Universiti Kebangsaan Malaysia. Satu set soalselidik yang dibanci sendiri &ldquo;Hospital Anxiety Depression Scale&rdquo; digunakan untuk mengukur tahap tekanan dan kebimbangan dikalangan wanita hamil yang berisiko tinggi. Dari sejumlah 38 responden,&nbsp; 16 (42.1%) wanita mengalami tahap kebimbangan yang ringan dan 22 (57.9%) mengalami tahap kebimbangan yang teruk. Tujuh belas (44.7%) wanita telah diklasifikasikan sebagai mengalami tekanan yang ringan dan 21 (55.3%) tekanan yang teruk.&nbsp; Kajian ini mendapati &ldquo;kurang informasi mengenai penyakit&rdquo;, &ldquo;faktor keluarga&rdquo; dan &ldquo;kewangan&rdquo; sebagai faktor-faktor yang mempengaruhi tahap tekanan dan bimbangan wanita hamil yang berisiko tinggi semasa berada diwad.&nbsp; Wanita hamil yang berisiko tinggi dalam kajian ini mengalami tekanan dan kebimbangan yang signifikan semasa berada dihospital. Dengan ini, ia adalah penting untuk jururawat menyedari dan peka pada faktor-faktor yang boleh menyebabkan tekanan dan kebimbangan supaya membolehkan ibu hamil yang berisiko tinggi bergembira dengan kehamilan dan kelahiran.</p>\r\n', 1),
(386, 383, '<p>\r\n	Objektif kajian ini adalah untuk menentukan prevalens senggugut dan kesannya terhadap aktiviti sekolah di kalangan remaja perempuan di sebuah sekolah menengah luar bandar di Daerah Hulu Langat, Selangor. Kajian rentas telah dijalankan di sebuah sekolah kerajaan. Responden kajian dipilih melalui kaedah persampelan rawak stratum dan terdiri daripada pelajar-pelajar Tingkatan 1 hingga 5. Mereka diberi borang soal-selidik yang mengandungi 20 soalan mengenai haid dan sosio-demografi. Tahap kesakitan senggugut diukur menggunakan skala numerikal. Seramai 300 pelajar terpilih untuk kajian ini dan prevalens senggugut dikalangan mereka ialah 62.3%. Penyakit senggugut mempunyai kaitan yang bermakna dengan kumpulan remaja pertengahan umur (15-17 tahun) (p=0.007), mereka yang mempunyai kitaran haid yang tetap (p=0.007) dan mempunyai sejarah keluarga yang positif (p=0.0001), tetapi tidak dengan umur bermulanya haid dan jangkamasa haid. Tahap kesakitan senggugut yang lebih tinggi pada seseorang pelajar mempengaruhi kadar keciciran sekolah (p=0.0005), daya tumpuan di dalam kelas (p=0.0005)&nbsp; dan penglibatan dalam sukan (p=0.008). Kajian ini menunjukkan prevalens yang tinggi di kalangan remaja perempuan dan memberi kesan negatif terhadap aktiviti sekolah mereka.</p>\r\n', 1),
(387, 384, '<p>\r\n	Tujuan penyelidikan ini ialah untuk mengkaji tahap fungsi seksual di kalangan kaum wanita Malaysia yang rendah serta tinggi kekerapan aktiviti seks mereka, di peringkat primer. Satu kajian irisan lintang yang melibatkan 230&nbsp; wanita Malaysia di peringkat primer telah dijalankan di klinik Bandar Tun Razak, Cheras. Soalselidik Indeks Fungsi Seks Wanita (Female Sexual Function Index) yang telah divalidasi ke Bahasa Melayu telah digunakan untuk menilai profil fungsi seksual wanita yang aktiviti seksnya rendah serta tinggi.&nbsp; Hampir setengah dari kaum wanita (44.3%) di dalam sampel kajian mengadakan hubungan seks&nbsp; 1 &ndash; 2 kali seminggu. Sementara hampir separuh daripada mereka (42.4%) mengadakan hubungan seks 1 &ndash; 2 kali sebulan atau kurang; dengan bilangan minoriti wanita (13.4%) mengadakan hubungan seks 3 &ndash; 4 kali seminggu.Wanita yang frekuensi seksnya rendah (SI Rendah) didapati megalami lebih banyak disfungsi seksual (&chi;&sup2;=28.98, p &lt; 0.001) berbanding dengan golongan wanita yang suka melakukan hubungan seks (SI Tinggi). Golongan wanita yang tidak suka melakukan seks ( frekuensi hubungan seks iaitu &le; 1 &ndash; 2 kali seminggu) didapati kurang terangsang dari segi seksual (&chi;&sup2;= 25.9, p&lt; 0.001), kurang menikmati orgasme (&chi;&sup2;=19.8, p&lt; 0.001), kurang mengeluarkan lendiran faraj sewaktu ghairah (&chi;&sup2;=11.1, p&lt; 0.001), mengalami lebih kesakitan ketika melakukan seks (&chi;&sup2;=4.3, p = 0.033) dan mengalami kepuasan seks yang amat kurang (&chi;&sup2;=12.6, p&lt; 0.001). Masalah disfungsi seksual di kalangan wanita amat memerlukan perhatian di peringkat penjagaan kesihatan primer. Penyelidik tidak pasti samada kekurangan seks atau masalah disfungsi seksual merupakan punca penyebab, tetapi hasil kajian ini amat penting untuk menggerakkan pakar perubatan melihat keperluan disfungsi seksual di kalangan wanita yang memerlukan bantuan.</p>\r\n', 1),
(388, 385, '<p>\r\n	Ketulan tiroid kerap ditemui tetapi neoplasma tiroid malignan&nbsp; jarang berlaku. Aspirasi sitologi&nbsp; jarum halus adalah kaedah diagnostik yang digunakan untuk menyaring pesakit tiroid yang memerlukan pembedahan. Kami mengkaji ketepatan diagnostik penggunaan&nbsp; aspirasi sitologi jarum halus sebagai kaedah modaliti&nbsp; diagnostik awal dalam penilaian klinikal ketulan tiroid. Sebanyak 2131 aspirasi jarum halus tiroid telah dilakukan di antara Januari 1995 dan Disember 2000 di Klinik FNA, Hospital Universiti Kebangsaan Malaysia (HUKM). Terdapat 441 (20.7%) kes-kes&nbsp; yang didapati tidak memuaskan untuk pemeriksaan sitologi manakala 1690 kes-kes yang selebihnya adalah memuaskan. Daripada jumlah ini, sebanyak 361 kes mempunyai diagnosis histopatologi. Korelasi sito-histopatologi dijalankan ke atas kes-kes ini. Keputusan kajian menunjukkan ketepatan diagnostik sebanyak 96.2% dengan kadar-kadar&nbsp; sensitiviti dan spesifisiti sebanyak 87.7% dan 98.4%. Nilai jangkaan positif adalah 93.4% dan nilai jangkaan negatif adalah 96.8%. Daripada keputusan kajian ini , kami merumuskan bahawa aspirasi jarum halus adalah kaedah diagnostik awal yang penting untuk penyiasatan ketulan tiroid.</p>\r\n', 1),
(389, 386, '<p>\r\n	Kanser ovari dan uterus merangkumi 5.0% dan 3.6% keseluruhan kes kanser wanita Malaysia. Kanser ovari (OC) merupakan ketumbuhan ginekologi pembawa maut yang utama kerana tiada terdapat gejala di peringkat awal sehingga kanser telah merebak. Ultrasonografi membenarkan pengesanan awal memberi peluang bagi pakar perubatan menyediakan rawatan dan kaunseling untuk wanita tersebut. Sebagai teras untuk saringan selektif, satu kajian telah dijalankan merangkumi 517 wanita bandar berusia 45+ tahun bebas penyakit, bukan pengguna HRT dan masih mempunyai uterus. Normogram ovari, uterus dan endometrium yang diperolehi daripada&nbsp; pemeriksaan ultrasound garisdasar dibentang.&nbsp; Sampel kajian terdiri daripada 58% dan 42% wanita pra dan pos-menopos dengan umur purata 51.27 &plusmn; 5.35 tahun. Lebih dua pertiga terdiri dari kaum Cina, diikuti oleh Melayu dan India. Kajian menunjukkan saiz purata jasad uterus dan ukuran endometrium (ET) adalah 7.21 &plusmn; 1.67x4.36 &plusmn; 1.30cm and 6.36 &plusmn; 3.73mm masing-masing. Saiz uterus wanita pre-menopos lebih besar berbanding pos-menopos (p&lt;0.0005) dan atrofi uterus berlaku berikutan umur dan menopos. Ukuran ET juga lebih tebal pada wanita pre berbanding pos-menopos (p&lt;0.0005), semakin tipis mengikut usia. Wanita pada awal pos-menopos menunjukkan ET &lt;5mm, mencapai 2mm pada wanita berusia &gt;70 tahun. Dengan demikian, tetapan normal klinikal kesemua wanita pos-menopos tanpa mengira kaum ditetapkan pada ET&lt;5mm. Hampir seperlima wanita (18.9%) didapati mempunyai fibroid uterus, sebanyak sesuku (25.0%) semasa pre berbanding 10% pos-menopos (p&lt;0.0005). Purata isipadu ovari (OV) kanan dan kiri masing-masing adalah 5.48 &plusmn; 7.85cm<sup>3</sup> and 5.80 &plusmn; 16.78cm<sup>3</sup>. Terdapat pengurangan OV mengikut usia dan menopos, daripada&nbsp; 7cm<sup>3</sup> pada usia 45-49 tahun kepada &lt;1.5cm<sup>3</sup> pada 70 tahun, menghasilkan tetapan normal klinikal wanita Malaysia pre dan pos-menopos pada OV &lt;7.5cm<sup>3</sup> and &lt;3.5cm<sup>3</sup>. Sista ovari tidak ada bahaya dijumpa pada 2.9% dengan saiz purata 65.14 &plusmn; 88.22cm<sup>3</sup>. Sebagai kesimpulan, normogram ET dan OV harus disediakan bagi populasi Malaysia untuk memudahkan saringan kanser ginekologi.</p>\r\n', 1),
(390, 387, '<p>\r\n	Merokok dikalangan remaja adalah suatu masalah kesihatan yang perlu diatasi kerana ia mengakibatkan berbagai penyakit kronik dan meningkatkan mortaliti. Ia juga berkaitan rapat dengan tabiat-tabiat berisiko tinggi lain di kalangan remaja. Ini adalah satu kajian keratan rentas untuk menentukan prevalens merokok di kalangan pelajar-pelajar lelaki Tingkatan 4 dan 5 dan&nbsp; sebab-sebab mereka merokok. Pelajar- pelajar dari tiga buah sekolah menengah di Daerah Hulu Langat di pilih secara rawak. Mereka yang bersetuju mengambil bahagian diberi borang soal-selidik. Sejumlah 343 pelajar terpilih dan prevalens merokok&nbsp; dikalangan mereka ialah 37%. Walaupun majoriti daripada mereka mula merokok dalam lingkungan umur 13-15 tahun, 21% daripada mereka mula merokok dibawah umur 12 tahun (semasa sekolah rendah). Terdapat perhubungan signifikan antara merokok dikalangan remaja dan ahli keluarga mereka yang merokok (p&lt;0.05). Dua sebab utama mereka mula merokok ialah &lsquo;sikap ingin tahu&rsquo; (69%) dan pengaruh kawan (51%). Walaubagaimana pun sebab utama mereka meneruskan tabiat merokok ialah &lsquo;tekanan mental&rsquo; (70%) dan ketagihan (49%). Faktor utama yang menyebabkan mereka menghadapi tekanan mental adalah &lsquo; kerja sekolah&rsquo;. Kesimpulannya,&nbsp; program anti-merokok perlu di mulakan awal di sekolah rendah dan remaja-remaja perlu diajar cara-cara menghadapi tekanan mental dan menyelesaikan masalah dengan efektif.</p>\r\n', 1),
(391, 388, '<p>\r\n	Kecederaan tajam dikalangan pekerja kesihatan di HUKM telah mendapat perhatian yang serius di kebelakangan ini kemungkinan kerana wujudnya penyakit-penyakit seperti HIV, Hepatitis B dan Hepatitis C yang boleh membawa kesan buruk kepada yang tercedera. Matlamat kajian ini ialah untuk memperolehi kefahaman yang lebih mendalam terhadap masalah emosi yang dialami oleh pekerja-pekerja kesihatan yang telah dicedera oleh benda tajam dan strategi yang diguna untuk mengatasi masalah tersebut. Satu soal selidik separa struktur yang mengandungi 28 bahan telah direkabentuk oleh penyelia dan penyelidik kerana soal selidik tidak wujud untuk di guna pakai diatas sebab tiada kajian serupa dijalankan sebelum ini. Kumpulan sasaran terdiri daripada 64 pekerja kesihatan dalam pelbagai kategori yang bertugas di HUKM. Data telah dikutip dalam tempoh lima bulan dari November 2000 hingga Mac 2001. Hasil kajian menunjukkan bahawa kumpulan yang paling banyak tercedera ialah jururawat terlatih, 90% daripada mereka merasa sedih dan risau diatas kemungkinan mendapat penyakit . Didapati tiada perbezaan signifikan dalam perasaan stres dan setiap kategori mengguna strategi berlainan untuk mengatasi tragedi ini. Komen dan pendapat terbuka yang disuarakan oleh responden mengenai pencegahan dan pengendalian kecederaan tajam di HUKM telah diberi perhatian. Beberapa pendapat daripada responden telah dikumpul dan dibincang. Akhirnya beberapa implikasi kejururawatan dan syur telah dikemukakan.</p>\r\n', 1),
(392, 389, '<p>\r\n	Satu kajian prevalens stres pekerjaan telah dilakukan dikalangan 185 eksekutif dan pengurus di ibu pejabat syarikat tembakau antarabangsa. Objektif kajian ialah untuk mengenalpasti prevalens stres pekerjaan dan faktor-faktor penyebab stres yang dialami pekerja ini. Soal-selidik daripada &lsquo;Personal Stress Inventory&rsquo; oleh O&rsquo; D Donell (1984) telah digunakan. Data berkaitan faktor sosiodemografi, simptom stres dan faktor stres berkaitan dengan polisi organisasi, stuktur&nbsp; organisasi dan sekitaran kerja telah dikumpulkan. Hasil kajian mendapati prevalens stress dikalangan eksekutif sebanyak 68.1% dan pengurus eksekutif sebanyak 67.9%, perbezaan ini tidak bererti secara statistiks. Faktor sosioekonomi yang bererti secara statistik berkait dengan stres ialah pendapatan, bilangan anak dan faktor personal. Faktor stres tempat pekerjaan yang bererti secara statistik ialah kurang penghargaan kerja, terlalu fokus kepada kualiti kerja, beban kerja yang banyak dan bekerja jangkamasa yang panjang.</p>\r\n', 1),
(393, 390, '<p>\r\n	Penyakit Kimura adalah penyakit benigna dan kronik yang puncanya tidak diketahui. Ia biasanya berlaku pada pesakit berbangsa Oriental yang datang dengan ketumbuhan di bawah kulit di bahagian kepala dan leher yang boleh disalahdiagnosa sebagai tumor malignan yang memerlukan pembedahan radikal dan kemoterapi intensif. Ia telah dilaporkan di dalam makalah berbahasa China dan Jepun. Ia sukar didiagnosa sebelum laporan biopsi tisu manakala sitologi aspirasi jarum (<em>fine needle aspiration cytology</em>) juga tidak banyak membantu.&nbsp; Justeru itu, jika pakar patologi kurang berpengetahuan tentang penyakit ini, ianya akan disalah anggap sebagai lesi yang malignan. Kami laporkan di sini satu kes pesakit Melayu yang datang dengan ketumbuhan parotid yang pada mulanya didiagnos sebagai limfoma Hodgkin. Penyakit Kimura sepatutnya disyaki apabila seorang pesakit lelaki muda dari Asia yang datang dengan ketumbuhan di bahagian kepala dan leher yang tidak sakit, dan disertai oleh peningkatan sel eosinofil (<em>hypereosinophilia).</em></p>\r\n', 1),
(394, 391, '<p>\r\n	Ini adalah kajian terkawal rawak rabun berganda prospektif, untuk membandingkan keadaan intubasi pada 60 saat dengan rocuronium sebanyak 0.6 mg/kg menggunakan tiga teknik induksi: propofol-placebo(PP), propofol-ephedrine(PE) dan propofol-placebo crystalloid(PC).Sembilan puluh pesakit telah dipilih, dan menerima secara rawak salah satu daripada teknik di atas. Induksi dilakukan dengan memberikan fentanyl 2 &micro;g/kg, diikuti dengan sama ada propofol 2.5 mg/kg dan normal saline plasebo (kumpulan PP dan PC) atau propofol 2.5 mg/kg dan ephedrine 70 &micro;g/kg (kumpulan PE) dalam masa 30 saat. Selepas itu rocuronium 0.6 mg/kg diberikan dan intubasi trakea dilakukan selepas 60 saat. Keadaan intubasi boleh diterima secara klinikal dalam semua pesakit kecuali empat pesakit dari kumpulan propofol-placebo. Kadar intubasi yang terbaik adalah paling tinggi dalam kumpulan PE (94%) diikuti oleh kumpulan PC (81%)dan terendah pada kumpulan PP (50%)<strong>. </strong>Kesimpulannya, induksi dengan menggunakan propofol-ephedrine dan propofol-placebo-crystalloid memberikan keadaan intubasi yang lebih baik daripada propofol-placebo apabila rocuronium 0.6 mg/kg digunakan untuk intubasi pada 60 saat.</p>\r\n', 1),
(395, 392, '<p>\r\n	Resusitasi kardiopulmonari (CPR) adalah penting dan pegawai perubatan siswazah (PPS) patut mahir menjalankannya. Para PPS yang baru sahaja selesai pengajian mereka ditugaskan ke wad perubatan akut&nbsp; dan wad pembedahan akut. Sekiranya pesakit di wad mengalami kematian mengejut akibat serangan jantung, merekalah yang akan mula merawat pesakit tersebut. Oleh itu seseorang PPS harus yakin dengan kemahirannya untuk menjalankan CPR sekiranya perlu. Kami telah menjalankan kajian berkaitan pengetahuan menjalankan CPR ke atas 26 PPS baru di Hospital Universiti Kebangsaan Malaysia (HUKM). Perkara-perkara yang dianalisis termasuk: tanggapan para PPS itu sendiri berkaitan kemahiran, pengetahuan dan tahap keyakinan mereka dalam menjalankan CPR. Kami menilai pengetahuan menggunakan satu set soalan berkaitan resusitasi kadiopulmonari. Hasil kajian mendapati bahawa 16/26 (61.5%) daripada PPS, menilai diri mereka sebagai mempunyai tahap pengetahuan yang tidak mencukupi manakala 46.2% tidak mempunyai keyakinan dalam menjalankan CPR. Kebanyakan mereka mendapat markah rendah dalam keputusan ujian penulisan, di mana min adalah 5.7 <u>+</u> 1.8. Tujuh daripada 26 (27.0%) PPS meletakkan tangan mereka pada posisi yang salah ketika menjalankan CPR. Hanya 4 dan 9 daripada 26 PPS meletakkan pad sternum dan pad kardiak pada posisi yang betul. Kami membuat kesimpulan bahawa pengetahuan, tanggapan kemahiran dan tahap keyakinan PPS dalam menjalankan CPR perlu diperbaiki dan perlu penilaian yang lebih mendalam. Institusi Pengajian Perubatan perlu meneliti kembali kurikulumnya supaya dapat menyediakan PPS agar lebih bersedia menghadapi situasi kecemasan.</p>\r\n', 1),
(396, 393, '<p>\r\n	Program Jagarawatan Di Rumah (HCNP) di HUKM dimulakan dalam tahun 1998 dan penilaian program ini diperlukan. Tujuannya ialah untuk menilai keberkesanan Program Jagarawatan Di Rumah ke atas rehabilitasi strok di HUKM. Satu kajian retrospektif telah dijalankan. Satu rekabentuk padanan pasangan (matched pair) ke atas 69 orang pesakit strok yang dimasukkan ke HUKM dari bulan Ogos 1998 hingga Disember 2000 disediakan. Peserta kajian terdiri dari kumpulan HCNP (n=35), dan <em>Non-HCNP</em> (n=34). Padanan dibuat berdasarkan lima kriteria; umur, jantina, bangsa, tarikh kemasukan, dan keterukan ketidakupayaan pesakit. Kumpulan HCNP telah dikenalpasti melalui data yang diperolehi dari unit HCNP dan rekod perubatan mereka dikesan. Bagi kumpulan <em>Non-HCNP</em> pula, peserta dikenalpasti berdasarkan kriteria yang sama dengan kumpulan HCNP. Pada kriteria yang dikenalpasti, tidak terdapat perbezaan yang signifikan antara kedua-dua kumpulan. Aktiviti kehidupan harian (ADL) diukur menggunakan &lsquo;Modified Barthel Index&rsquo; sebanyak dua kali; semasa kemasukan (berasaskan rekod) dan selepas strok. Uijan-t digunakan untuk data parametrik dan khi-kuasa dua bagi data non-parametrik untuk mengesan hubungan signifikan skor antara dua kumpulan. Hasil kajian menunjukkan tidak terdapat perbezaan signifikan (p=0.1990) dalam aktiviti kehidupan harian (ADL) semasa kemasukan dan pos strok ADL antara kedua kumpulan. Walaubagaimana pun, terdapat perbezaan signifikan (HCNP p=0.000 dan Non-HCNP, p=0.000) dalam ADL semasa kemasukan dan pos strok bagi pesakit yang mendapatkan rawatan alternatif jika dibandingkan dengan pesakit yang tidak mendapat rawatan alternatif.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(397, 394, '<p>\r\n	Salah satu faktor risiko penting dalam kanser serviks adalah jangkitan virus papiloma manusia (<em>human papillomavirus </em>- HPV). Gen p53 merupakan salah satu sasaran utama gen E6 HPV. Protein E6 mempunyai keupayaan untuk merangsang degradasi p53, menghentikan beberapa fungsi p53 jenis-liar dan bersaing dengan fungsi lain p53 seperti mencegah pertumbuhan kanser. Tujuan kajian ini adalah untuk mengenalpasti perbezaan pengekspresan p53 dalam neoplasia serviks pra-malignan dan malignan. Kajian retrospektif ini melibatkan 100 kes neoplasia serviks [21 neoplasia serviks intraepitelial 1 (<em>cervical intraepithelial neoplasia</em> 1- CIN1), 8 CIN2, 25 CIN3, 36 karsinoma sel skuamus, 7 adenokarsinoma dan 3 adenoskuamus karsinoma). Semua kes dikaji dengan kaedah immunohistokimia menggunakan antibodi monoklonal p53. Tiga puluh enam daripada 54 kes pra-malignan (66.7%) adalah positif terhadap protein p53 berbanding dengan 40 daripada 46 kes malignan (87.0%) yang menunjukkan kepositifan. Dua puluh sembilan daripada 54 (53.7%) kes CIN menunjukkan sama ada pengekspresan fokal atau tiada pengekspresan. Sebaliknya, 84.8% (39/46) kes karsinoma invasif menunjukkan pengekspresan regional atau pengekspresan keseluruhan. Pengekspresan p53 didapati lebih tinggi dalam neoplasia serviks malignan berbanding lesi serviks pra-malignan. Ini mencadangkan bahawa pengekspresan p53 bukan satu fenomena awal dalam patogenesis kanser serviks. Kajian ini juga menunjukkan terdapat peningkatan peratusan pengekspresan p53 dalam CIN2 dan CIN3 berbanding dengan CIN1. Akan tetapi, terdapat beberapa kes yang tiada pengekspresan p53 mencadangkan kemungkinan penglibatan faktor-faktor lain dan kajian HPV lanjutan adalah diperlukan.</p>\r\n', 1),
(398, 395, '<p>\r\n	Rawatan otitis media kronik yang betul boleh mengurangkan insiden abses otogenik otak. Objektif kajian ini adalah untuk menilai profil klinikal, rawatan dan kesan pesakit yang didiagnos dengan abses otogenik otak. Rekod perubatan pesakit di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) yang dirawat sebagai abses otogenik otak dari Januari 1997 sehingga Januari 2006 dianalisa secara retrospektif. Dalam tempoh&nbsp; tersebut seramai lebih kurang 10,800 pesakit mengalami jangkitan telinga tengah kronik mendapat rawatan susulan di Klinik Telinga Hidung dan Tekak, PPUKM. Sepuluh pesakit (2 perempuan, 8 lelaki) dengan purata umur 47 tahun (umur di antara 11 sehingga 69 tahun). Min bagi jangka masa rawatan susulan adalah 14 bulan. Semua pesakit menghidapi kolesteatoma. Kesemua pesakit mempunyai sejarah discaj telinga yang kronik, sakit kepala, sakit telinga dan demam. Enam dari 10 pesakit&nbsp; mempunyai abses serebelar dan 4 mempunyai abses lobar temporal. Kesemua pesakit dirawat dengan antibiotik spektrum luas. Lima pesakit menjalani pembedahan mastoidektomi sebagai rawatan utama manakala abses otak dirawat secara konservatif. Manakala lima pesakit lagi menjalani pembedahan kraniotomi dan pengaliran nanah diikuti dengan pembedahan mastoid apabila fungsi neurologi sudah stabil. Tiada pesakit yang mempunyai tanda serebelar yang kekal ketika rawatan susulan dan tiada kes kematian. Di sini kami menunjukkan bahawa rawatan awal dan tepat bagi pesakit abses otogenik otak memberikan hasil pemulihan neurologi yang baik.</p>\r\n', 1),
(399, 396, '<p>\r\n	Plasenta praevia adalah masalah semasa hamil yang boleh mendatangkan komplikasi terhadap ibu dan janin.Penyelidikan yang dijalankan ini bertujuan untuk menilai pengetahuan tentang&nbsp; plasenta praevia dikalangan wanita hamil. Kajian keratan rentas dijalankan di Hospital Ipoh, Perak dikalangan 323 orang&nbsp; pesakit antenatal and posnatal. Parameter sosio demografi (seperti umur, keturunan , bilangan kandungan, pekerjaan, dan tahap pengajian), dan sejarah plasenta praevia telah dikaji dalam hubungkait dengan tahap pengetahuan dan tabiat terhadap plasenta praevia. Seramai 20 orang (6.2%) dari 323 wanita tersebut mengidap atau mempunyai sejarah plasenta praevia. Tiga diantara mereka mempunyai sejarah and 17 lagi mengidap plasenta praevia ketika itu dengan prevalens sebanyak 5.3%. Markah purata pengetahuan yang diperolehi oleh peasakit adalah 11.8 menandakan tahap pengetahuan yang rendah. Hanya pekerjaan, tahap pendidikan dan sejarah plasenta praevia didapati mempunyai hubungkait dengan tahap pengetahuan tentang plasenta praevia diantara semua pesakit obstetrik. Terdapat hubungkait yang ketara mengenai tabiat pesakit yang mengidapi plasenta praevia sekarang serta pesakit yang telah mempunyai sejarah plasenta praevia terhadap tahap pengetahuan mengenai plasenta praevia (p=0.037, &lt;0.05). Sebagai rumusan, pengetahuan serta tabiat terhadap plasenta praevia dikalangan pesakit obstetrik di Hospital Ipoh lebih baik dikalangan mereka yang berpendidikan tinggi, mempunyai pekerjaan &lsquo;kolar puteh&rsquo;&nbsp; dan pesakit yang mempunyai atau pernah mengidap plasenta praevia.</p>\r\n', 1),
(400, 397, '<p>\r\n	Persekitaran tempat kerja di kalangan jururawat unit rawatan intensif (ICU) menyumbang kepada punca stres. Ramai penyelidik telah menjelaskan bahawa stres di unit rawatan rapi adalah disebabkan oleh masalah penjagaan pesakit yang rumit dan penggunaan teknologi yang canggih. Kajian ini bertujuan mengenalpasti&nbsp; prevalen stres di kalangan jururawat yang berkerja di ICU, Hospital Universiti Kebangsaan Malaysia (HUKM) dan faktor-faktor yang mempengaruhi stres, dan meninjau&nbsp; tanda-tanda stres yang dialami. Kajian deskriptif ini dijalankan keatas 67 jururawat yang berkerja di ICU, HUKM. Data diperolehi melalui soalselidik yang diedarkan sendiri oleh penyelidik. Soal selidik terdiri daripada maklumat demografi, faktor-faktor penyumbang stres dan pengalaman stres yang dialami. Data dianalisis dalam bentuk frekuensi dan peratusan. Ujian &ldquo;Chi-square&rdquo; telah digunakan untuk melihat hubungan antara sosio-demografi dengan faktor-faktor yang menyumbang kepada stres. Keputusan kajian mendapati 100% atau semua&nbsp; jururawat di ICU mengalami stres. Pengetahuan, pengalaman kerja, keadaan pesakit yang kritikal dan persekitaran kerja adalah antara faktor-faktor yang menyumbang kepada kejadian stres di kalangan jururawat, ICU, HUKM. Langkah-langkah seperti menggalakkan jururawat ICU mengikuti kursus pos-basik ICU dan meningkatkan bilangan jururawat, boleh mengurangkan stres di kalangan jururawat ICU. Pihak pengurusan hospital juga memainkan peranan yang penting untuk mengurangkan stres di kalangan jururawat yang berkerja di ICU disebabkan stres boleh memberi kesan ke atas prestasi kerja dan kualiti jagarawatan.</p>\r\n', 1),
(401, 398, '<p>\r\n	Tradisi memusatkan faktor kosmetik dan fisikal sebagai model tanpa memberi perhatian pada isu psikososial adalah rintangan untuk mencapai kejayaan dalam penjagaan rawatan obesiti. Objektif artikel ini adalah untuk melapurkan satu kes mengenai seorang pesara askar berpangkat sarjen yang menunjukkan sindrom makan malam(SMM) dan obesiti morbid serta distimia, dan juga untuk membincangkan aspek psikobiologi kes disamping untuk menilai keberkesanan kombinasi rawatan farmakoterapi, dan terapi kognitif tingkahlaku serta kaunseling pemakanan. Diagnosis klinikal di buat menggunakan Temuduga Klinikal Berjadual (TKB) untuk diagnosis DSM-III-R dan tahap kemurungan dinilai dengan Skala Penarafan Kemurungan Hamilton. Indeks massa badan pesakit adalah 45. Beliau didapati mengalami Distimia dan Skor Skala Penarafan Kemurungan&nbsp; Hamilton adalah 13. Kami mendapati kombinasi farmakoterapi, terapi kognitif tingkahlaku dan pendidikan pemakanan serta bantuan pakar perubatan telah terbukti berkesan dalam rawatan Sindron Makan Malam (SMM), Obesiti Morbid dan Distimia.</p>\r\n', 1),
(402, 399, '<p>\r\n	Abses parafarinks adalah keadaan perubatan yang serius di mana ia boleh menyebabkan komplikasi yang mengancam nyawa. Insidens penyalit ini didapati berkurangan sejak penggunaan antibiotik. Kami melaporkan dua kes pengumpulan nanah di parapharyngeal di kalangan pesakit rendah tahap imuniti. Kami percaya bahawa diagnosis awal, rawatan antibiotik spektrum luas, pembedahan dan pengaliran nanah awal dapat mencegah komplikasi yang serius.</p>\r\n', 1),
(403, 400, '<p>\r\n	Adenoma pleomorfik (PA) ialah neoplasma epitelium kelenjar liur yang membesar perlahan-lahan dan biasanya didapati pada kelenjar liur parotid. Ia jarang didapati pada kelenjar liur submandibular terutama pada kumpulan umur pediatrik. Pembedahan adalah rawatan pilihan. Satu kes PA pada pesakit perempuan Melayu berumur 8 tahun dibentangkan. Pemeriksaan klinikal menunjukkan bengkakan di bahagian submandibular kanan berukuran 2x2sm. Diagnosis adenoma pleomorfik (PA) dibuat melalui sitologi sedutan jarum halus. Pesakit menjalani pembedahan submandibulektomi tanpa komplikasi. Dua tahun selepas pembedahan, pemeriksaan susulan menunjukkan pesakit masih asimptomatik.</p>\r\n', 1),
(404, 401, '<p>\r\n	Penyakit angin ahmar (strok) semakin menjadi masalah utama di dalam penjagaan kesihatan di negara kita disebabkan oleh umur populasi negara yang semakin meningkat. Keberkesanan rawatan strok di peringkat akut menyebabkan tiga daripada empat pesakit strok dapat melepasi peringkat akut strok. Doktor dan mereka yang terlibat di dalam penjagaan strok berpendapat bahawa penjagaan strok lanjutan bermula selepas setahun selepas serangan strok, memandangkan tempoh ini berhubungkait dengan kebarangkalian untuk terus hidup selepas mendapat serangan strok. Penjagaan strok lanjutan adalah kompleks, melibatkan keseluruhan aspek kehidupan pesakit; keperluan fisikal, psikologikal dan penglibatan ke dalam komuniti. Proses rehabilitasi yang merupakan tunjang utama penjagaan strok lanjutan seharusnya menumpukan kepada &lsquo;evidence-base&rsquo; untuk menjadi lebih effektif dan relevan kepada pesakit strok.</p>\r\n', 1),
(405, 402, '<p>\r\n	Kepuasan pesakit merupakan satu elemen kritikal yang menjadi minat kepada semua perawat perubatan. Ini adalah kerana pesakit yang berpuas hati akan mudah mendapatkan dan mengikuti segala arahan perubatan yang diberi. Objektif kajian ini adalah untuk mengenalpasti faktor-faktor yang mempengaruhi&nbsp; kepuasan pesakit terhadap perkhidmatan&nbsp; di Jabatan Perubatan Kecemasan HUKM (JPE HUKM). Kajian di JPE HUKM ini dilakukan dari Januari 2007 hingga Mac 2007. Satu kaedah pesampelan mudah (convenience sampling) telah dilakukan dimana 100 orang pesakit dari kumpulan &lsquo;triage&rsquo; 4 telah diambil. Kepuasan pesakit telah diukur menggunakan Skala Kepuasan Penjagaan Kecemasan Pelanggan Davis (Davis Consumer Emergency Care Satisfaction Scale) dengan sedikit pengubahsuaian. Ianya terdiri daripada 19 soalan yang mana setiap satu soalan diberi markah 1 hingga 5 mengikut skala Likert (1=sangat tak setuju dan 5=sangat setuju) keatas kepuasan pesakit terhadap jagarawatan di triage, kelakuan perawat perubatan di triage&nbsp; dan pendidikan kesihatan. Keputusan menunjukan 75 (75%) pesakit berpuas hati. Didapati terdapat tiada perbezaan&nbsp; yang signifikan antara pesakit lelaki dan perempuan dengan&nbsp; CESCC skor (t=0.308, p&gt;0.05). Hubungan korelasi yang positif&nbsp; didapati antara jumlah tahap kepuasan pesakit dan &lsquo;caring&rsquo; skor dan pendidikan kesihatan (r=0.905, p&lt;0.05) and teaching scores (r=0.695, p&lt;0.05). Secara keseluruhan kebanyakan pesakit berpuashati dengan perkhidmatan yang diterima di JPE HUKM. Kepuasan pesakit akan tetap menjadi satu ukuran penting dalam menentukan kualti perkhidmatan kecemasan di mana mana hospital.</p>\r\n', 1),
(406, 403, '<p>\r\n	Ini adalah kajian irisan lintang yang memeriksa kaitan kualiti hidup (KH) dengan faktor klinikal dan cara menangani stres pada pesakit Skizofrenia (N=92) dalam remisi daripada bulan Jun hingga Disember 2002. Tahap remisi ditentukan berdasarkan skala BPRS (Skala Ringkas Psikotik Psikiatri) dan diagnosa oleh Pakar Psikiatri yang merawat pesakit berdasarkan Temuduga Klinikal Berjadual Diagnosa untuk Manual diagnostik Statistik Edisi ke IV (DSM IV). Mereka berikutnya diminta melengkapkan soalan data demografik dan faktor klinikal serta seterusnya soalan pernilaian kualiti hidup(KH) dan cara tangani stress dengan masing-masing menggunakan 36 Item Soalan Ringkas Kajiselidik Kesihatan (SF36) untuk Kajian Hasil Perubatan dan Inventori Menangani Situasi Stres (CISS). KH pada terma kesihatan mental secara keseluruhannya pada pesakit Skizofrenia menunjukan ada perbedaan signifikan didalam kumpulan etnik (p&lt;0.05), status pekerjaan (p&lt;0.05), rawatan antipsikotik dan bilangan kemasukan pesakit ke wad (p&lt;0.05). Kaum Cina dan mereka yang bekerja mempunyai perbezaan signifikan (p&lt;0.05) pada KH untuk terma keseluruhan kesihatan mental. Cara menangani stress yang paling kerap digunakan oleh pesakit Skizofrenia adalah komponen menarik perhatian berorientasikan pengelakan. Menangani stres berorientasikan tugas mempunyai korelasi positif yang signifikan pada komponen mental KH. Sementara menangani berorientasikan emosi mempunyai korelasi negatif pada semua domin KH. Kesimpulannya, KH yang baik berkorelasi secara positif dengan cara menangani stres berorientasikan tugas dan berkorelasi secara terbalik pada cara menangani berorientasikan emosi.</p>\r\n', 1),
(407, 404, '<p>\r\n	Sindrom Turner adalah salah satu keabnormalan kromosom yang kerap berlaku pada bayi-bayi perempuan yang baru lahir. Lebih dari separuh pesakit yang menghidap sindrom Turner mempunyai kariotip 45X. Pesakit yang selebihnya menunjukkan keabnormalan struktur kromosom seks ataupun menunjukkan&nbsp; mozek dengan kromosom seks normal atau abnormal. Pesakit-pesakit mozek sindrom Turner yang mempunyai kromosom seks X yang kedua, kebiasaannya tidak menunjukkan sebarang tanda-tanda klinikal yang penting. Walaubagaimanapun, pesakit sindrom Turner yang mempunyai kromosom Y yang kedua atau bahan kromosom Y, berisiko menghadapi penyakit gonadoblastoma di kemudian hari. Tujuan kajian ini ialah untuk membandingkan teknik sitogenetik konvensional (kariotip) dan pendaflor <em>in situ</em> hibridisasi (<em>FISH</em>) di dalam mengesan bahan kromosom Y pada pesakit sindrom Turner. Kami juga membuat perbandingan darjah mozek yang dikesan diantara teknik kariotip sitogenetik dan penghibridan pendaflor (<em>FISH</em>). Kariotip dan <em>FISH</em> telah dijalankan ke atas lapan sampel darah dari pesakit sindrom Turner yang dikumpulkan diantara tahun 2004 dan 2006.&nbsp; Dua pesakit sindrom Turner dikesan mempunyai kawasan penentuan seks pada gen kromosom Y (SRY) dengan menggunakan teknik <em>FISH</em>. Kadar pengesanan kes-kes mozek sindrom Turner juga bertambah sebanyak 88% setelah menggunakan teknik <em>FISH</em>. Hasil kajian ini menunjukkan bahawa teknik <em>FISH</em> lebih efektif dari teknik kariotip di dalam pengesanan bahan kromosom&nbsp; serta penentuan&nbsp; darjah mozek Y pada pesakit sindrom Turner. Teknik <em>FISH </em>juga adalah cepat dan lebih kos efektif di dalam&nbsp; diagnosis sindrom Turner dalam penentuan darjah mozekisme.</p>\r\n', 1),
(408, 405, '<p>\r\n	Ini adalah suatu kajian cross-sectional pada 218 orang ibu mengandung di suatu hospital kerajaan di bandar. Kajian ini bertujuan menilai pengetahuan, sikap dan sumber maklumat berkenaan penyusuan susu ibu. Hasil kajian boleh digunakan untuk promosi penyusuan. Hampir semua responden (96.8%) berniat menyusui bayi. Kebanyakan daripada mereka (74.8%) berpengetahuan luas mengenai penyusuan iaitu&nbsp; kolostrum and susu ibu adalah makanan terbaik; ia melindungi bayi daripada penyakit dan alahan; mengenyangkan; membantu perkembangan gigi bayi dan penyembuhan ibu selepas bersalin; merapatkan jalinan; senang didapati dan menjimatkan. Terdapat dua tanggapan salah iaitu peyusuan patut diberhentikan jikalau ibu atau bayi jatuh sakit, dan pemberian air kepada bayi yang menerima penyusuan ibu sepenuh untuk mengelakkan dari penyahhidratan. Kebanyakan ibu (83.9%)memberikan respons positif kepada penyusuan ibu: penggunaannya lebih mudah dari formula bayi; tiada kesan negatif kepada perhubungan perkahwinan atau penjagaan keluarga; akan memulakan penyusuan sebaik sahaja bayi dilahirkan; bersetuju dengan pelarangan susu botol dan puting di hospital dan mereka tidak akan berhenti penyusuan ibu walaupun dihalangi suami. Hanya 56.9% para ibu percaya mereka sanggup memberi penyusuan ibu di mana-mana sahaja. Sumber utama maklumat mengenai penyusuan ibu ialah daripada media massa (34.9%),kelas antenatal (32.1%)dan ibu lain yang berpengalaman.</p>\r\n', 1),
(409, 406, '<p>\r\n	Sebuah kajian prevalen telah dilaksanakan terhadap 79 orang operator telefon wanita yang berkhidmat di sebuah Pusat Telekomunikasi di Kuala Lumpur untuk menentukan prevalen penyakit anggota atas berkaitan kerja dan faktor-faktor risikonya. Pengumpulan data kajian telah dilaksanakan dari bulan Disember 2000 sehingga bulan Mei 2001. Soal selidik dan pemeriksaan fizikal telah digunakan untuk menentukan gred penyakit ini. Kadar respon untuk kajian ini adalah 94.9% (n=75) dan hasil menunjukkan bahawa prevalen penyakit anggota atas berkaitan kerja di kalangan operator telefon adalah 48.0%: Peringkat 1= 38.9%, Peringkat 2= 19.4%, Peringkat 3= 38.9%, Peringkat 4= 0% and Peringkat 5= 2.8%. Didapati bahawa subjek yang menghidapi penyakit ini mempunyai min tempoh berkhidmat di pusat telekomunikasi tersebut selama 11.59&plusmn;9.09 tahun, manakala subjek yang tidak menghidapi penyakit ini, min tempoh berkhidmat adalah selama 9.89&plusmn;8.48 tahun. Faktor-faktor yang dikaji seperti Indeks Jisim Tubuh, umur, etnik, kitaran haid, pengambilan terapi hormon, tempoh perkhidmatan di dalam unit dan di pusat telekomunikasi tidak berbeza di antara subjek yang menghidapi dan tidak menghidapi penyakit anggota atas berkaitan kerja. Ini mungkin disebabkan oleh saiz sampel yang kecil atau kesan &lsquo;healthy worker&rsquo; yang boleh dilihat di dalam kajian kesihatan pekerjaan. Hasil kajian ini menunjukkan bahawa hampir 40.0% dari pekerja mengalami simptom sakit dan neurologikal. Memandangkan tiada dari faktor yang dikaji mempunyai hubungan dengan penyakit anggota atas berkait pekerjaan, kajian dengan reka bentuk kawalan kes diperlukan untuk membolehkan kes dipadankan dengan kawalan yang sesuai supaya faktor pembaur boleh dikawal. Ini untuk memastikan faktor risiko yang berkaitan dengan anggota atas di kalangan pekerja Malaysia dapat dikenalpasti.</p>\r\n', 1),
(410, 407, '<p>\r\n	Pesakit yang memerlukan rawatan di Unit Rawatan Intensif berkemungkinan melalui pengalaman yang tidak menyenangkan. Pengalaman ini termasuk dalam bentuk insiden-insiden berfakta dan juga gangguan ingatan terhadap persekitaran dalam unit, sebagai contohnya, kerap bermimpi, halusinasi dan pengalaman yang menakutkan. Tujuan kajian ini adalah untuk mengenalpasti pengalaman yang dilalui pesakit ketika mereka dirawat dengan bantuan pernafasan mekanikal di ICU HUKM. Kajian yang berbentuk irisan rentas ini menggunakan soalselidik pengalaman rawatan intensif &ldquo;Intensive Care Experience Questionnaire&rdquo; yang mengandungi empat domain: kepekaan terhadap keadaan sekeliling, pengalaman yang menakutkan, pengingatan semula pengalaman dan kepuasan terhadap jagarawatan. Seramai 45 responden yang di unit rawatan intensif (ICU) memenuhi kiteria yang telah ditetapkan menyertai kajian ini. Kajian ini telah dijalankan di ICU HUKM dari Januari 2006 hingga Mac 2006. Kajian menunjukkan bahawa 20 responden (44%) adalah peka terhadap persekitaran mereka, 31 responden (69%) melaporkan pengalaman yang menakutkan. Kebanyakan responden, 43 (96%) berpuas hati dengan jaga-rawatan. Terdapat kolerasi yang positif antara kepekaan terhadap persekitaran dengan keupayaan responden untuk mengingat semula pengalaman (p&lt;0.05). Kepekaan pesakit terhadap persekitaran merupakan faktor sumbangan yang signifikan dalam menjangka keupayaan pesakit untuk mengingat kembali pengalaman semasa di ICU dengan nilai pekali beta&nbsp; 0.353 dan p&lt;0.05. Hasil kajian ini membolehkan jururawat memperlihatkan keperluan pesakit yang bernafas dengan bantuan mekanikal daripada maklumat pesakit. Pesakit melaporkan frustasi dalam usaha mereka untuk menyuarakan laporan pengalaman yang tidak menyenangkan masing-masing. Informasi ini memperihalkan paras frustasi sebenar yang dialami oleh pesakit apabila mereka ingin menyuarakan diri semasa dibantu oleh alat pernafasan mekanikal.</p>\r\n', 1),
(411, 408, '<p>\r\n	Keupayaan bertutur dengan pesakit dan saudara-mara tidak wujud secara semulajadi, lebih sukar lagi bagi doktor muda untuk meletakkan diri dalam situasi pesakit yang rumit. Satu kajian &lsquo;keratan rentas&rsquo; telah dilakukan untuk mengenalpasti kebolehan berkomunikasi graduan tahun 2004 Fakulti Perubatan, UKM semasa mereka menjalani posting sebagai &lsquo;pegawai perubatan siswazah&rsquo; di hospital seluruh Malaysia. Borang maklumbalas yang piawai digunakan untuk mengumpul data. Lima puluh sembilan peratus graduan mengakui mereka mempunyai kemahiran komunikasi yang sangat baik dengan pesakit, manakala 69% graduan mengakui mempunyai kemahiran komunikasi yang sangat baik dengan kakitangan sokongan dan 88% dengan rakan sebaya. Walaubagaimanapun, 38% dan 41% mengakui mereka mempunyai kemahiran komunikasi yang baik dengan pihak atasan dan keluarga pesakit, masing-masing. Hanya 22% daripada graduan mengakui bahawa mereka mempunyai kemahiran yang baik di dalam menyampaikan berita buruk, manakala 50% dan 81% mengakui mereka mempunyai kemahiran kaunseling pesakit dan pengambilan persetujuan untuk prosedur, masing-masing. Oleh itu disarankan perancang kurikulum memberi penekanan yang sewajarnya dalam aspek kemahiran komunikasi untuk bakal doktor di masa akan datang.</p>\r\n', 1),
(412, 409, '<p>\r\n	Kanser payu-dara dan kanser serviks merangkumi 30.4% dan 12% kesemua kes kanser di Malaysia. Oleh demikian saringan kanser organ reproduktif pada ketika wanita mendekati menopaus adalah penting. Kajian ini melapurkan hasil penilaian kemasukan 495 wanita kawasan bandar Malaysia berusia 45 tahun ke atas yang telah mengambil bahagian dalam projek gayahidup sihat secara sukarela. Mereka terdiri daripada 58.0% wanita premenopaus dan 42.0% posmenopaus dengan min umur 51.27&plusmn;5.35 tahun. Lebih dua pertiga terdiri dari kaum Cina diikuti oleh Melayu dan India. Pada keseluruhan, calitan Pap abnormal didapati pada 7.6% iaitu 1.3% <em>cervical intraepithelial neoplasia</em> (CIN), 6.1% infeksi <em>human papilloma virus</em> (HPV) dan 0.2% <em>atypical squmous cells of undetermined significances </em>(ASCUS). Infeksi yis dan lain organisma dijumpai pada 6.9% dan 1.9% masing-masing. Wanita posmenopaus menunjukkan 2.8 kali ganda insiden kanser serviks sementara wanita premenopaus mempunyai kadar infeksi yang lebih tinggi, 11.8% lawan 4.7% (p=0.024). Kadar infeksi HPV adalah setara pada kedua-dua kumpulan. Kajian ini mendapati 1.3% mengidap kanser payu-dara, 3.6% memerlukan biopsi dan 3.4% perlu pemantauan berkala. Wanita posmenopaus memiliki lebih mamogram abnormal (p&lt;0.0005) jenis merbahaya yang memerlukan pemantauan. Akan tetapi kadar kanser payu-dara adalah lebih rendah pada posmenopaus berbanding kepada premenopaus, 0.010 lawan 0.015. Hasil kajian ini telah mengenalpasti saringan aktif kanser serviks dikalangan wanita posmenopaus kerana kadar kanser adalah lebih tinggi. Untuk mengesan perubahan abnormal payu-dara, proses saringan harus bermula lebih awal, di kalangan wanita pre- dan posmenopaus yang berusia 45 tahun ke atas.</p>\r\n', 1),
(413, 410, '<p>\r\n	Limfoma sel besar anaplastik (ALCL) adalah tumor yang jarang berlaku. Limfoma ini terdiri daripada kira-kira 3% daripada limfoma bukan-Hodgkin dewasa. Kerapkali ALCL sistemik primer melibat pada kedua-dua nodus limfa dan tapak ekstranodus. Seorang wanita berumur 44 tahun menampilkan dengan massa padat di bahagian fosa iliak kiri. Penemuan <em>ultrasound</em> menunjukkan massa tisu lembut <em>inhomogenous</em> yang agak berbatas jelas, berukuran 4x4x2.6cm di bahagian subkutanus dalam. Pemeriksaan histopatologi menunjukkan sel limfoid yang besar dengan nukleus yang atipikal, termasuk nukleus berbentuk ginjal. Sel neoplastik ini mengekspres ALK (pewarnaan pada kedua-dua sitoplasma dan nuklues), CD30 dan EMA tetapi penanda bukan pada sel-T (CD45RO dan CD3) dan sel-B (CD20 dan CD79&alpha;). Analisis hibridisasi in situ fluoresens (<em>FISH</em>) menunjukkan translokasi kromosom, t(2;5)(p23;q35). Berikutnya pesakit mengalami kesesakan nafas yang mendadak dan skan tomografi berkomputer torasik (CT) menunjukkan massa mengelilingi bronkus lobus atasan kanan. Pesakit juga mempunyai nodus limfa aksilari bilateral, berukuran 1 sm diameter (biopsi tidak dilakukan). Kawasan mediastinum dan endobronkus tidak menunjukkan sebarang keabnormalan. Pesakit menerima rawatan kemoterapi CHOP sebanyak 6 pusingan (<em>cycle</em>) dan pesakit ini sembuh daripada penyakit ini. ALCL jarang menampil sebagai tumor tisu lembut dan adalah disyorkan supaya penyakit ini dimasukkan dalam senarai sebagai diagnosis diferensial pada sebarang massa tisu lembut.</p>\r\n', 1),
(414, 411, '<p>\r\n	Peningkatan kos dan kesedaran masyarakat mengenai penyampaian kualiti perkhid&shy;matan yang tinggi telah memaksa pembekal dan pengurus jagaan kesihatan untuk menyiasat sistem pemberian jagaan kesihatan. Carta-alir klinikal (CK) telah diperke&shy;nalkan di banyak hospital dan telah diterima sebagai satu alat yang berfaedah dalam membantu organisasi jagaan kesihatan di seluruh dunia. Tambahan, CK juga dapat membekalkan perkhidmatan yang berkualiti tinggi secara berterusan dan dapat meng&shy;koordinasikan perkhidmatan kesi&shy;hatan dengan penggunaan sumber-sumber yang rendah. Telah dibuktikan sebagai alat yang berfaedah di negara-negara lain, CK se&shy;dang diperkenalkan dalam sistem jagaan ke&shy;sihatan di Malaysia. Tujuan artikel ini adalah untuk mengenengahkan faedah-faedah CK dalam meningkatkan kualiti jagaan kesihatan dan mengawal kos perubatan. Artikel-artikel yang berkaitan telah disemak. Kesimpulannya, sebahagian besar artikel-artikel yang di&shy;semak menyimpulkan terdapat kesan-kesan positif dalam mengimplementasikan CK. CK telah didapati dapat menu&shy;runkan dengan signifikannya jangka masa tinggal di hospital dan kos perubatan. Pengenalan <em>evidence based medicine</em>, hasil klinikal, audit klinikal, komuni&shy;kasi jabatan-jabatan, kerjasama dan perancangan penjagaan juga dipertingkatkan oleh CK. Cabar&shy;an kepada pembekal dan pengurus jagaan kesihatan adalah penglibatan dan komit&shy;men sepenuhnya dalam pembentukan dan implementasi CK untuk meningkatkan kua&shy;liti jagaan kesihatan dan kawalan kos.</p>\r\n', 1),
(415, 412, '<p>\r\n	Stres oksidatif dikaitkan dengan osteoporosis pos-menopaus yang meningkatkan ri&shy;siko patah tulang. Tokotrienol sawit ialah antioksidan yang poten dan berpotensi untuk digunakan bagi merawat osteoporosis pos-menopaus. Tujuan kajian ialah untuk me&shy;nentukan samada suplementasi tokotrienol sawit boleh mengurangkan stress oksidatif dan meningkatkan kekuatan tulang pada model tikus terovariektomi. Tikus dibahagi&shy;kan kepada empat kumpulan : (i) bedah sham (SHAM) (ii) kawalan ovariektomi (OVX) (iii) ovariektomi dan diberi 60mg/kg &alpha;-tokoferol melalui gavaj oral (OVX+ATF) (iv) ova&shy;riektomi dan diberi 60mg/kg tokotrienol sawit melalui gavaj oral (OVX+PTT). Selepas rawatan selama lapan bulan, sampel darah diambil untuk mengukur status oksidatif (MDA, SOD dan GPX) sementara tulang femur diuji kekuatan dan resistan biomeka&shy;nikal terhadap kepatahan. Ovariektomi menyebabkan stres oksidatif seperti yang di&shy;tunjukkan dengan peningkatan aras MDA dan penurunan aras aktiviti GPX. Tokotrie&shy;nol sawit menunjukkan perlindungan terhadap stres oksidatif aruhan ovariektomi se&shy;perti yang ditunjukkan dengan penurunan aras MDA dan peningkatan aktiviti GPX dan SOD pada kumpulan OVX+PTT. Apabila dibandingkan, &alpha;-tokoferol hanya berupaya meningkatkan SOD tetapi tidak setinggi tokotrienol sawit. Ujian biomekanikal menun&shy;jukkan bahawa ovariektomi tidak memberi kesan signifikan kepada kekuatan tulang selepas rawatan selama lapan minggu. Suplementasi tokotrienol sawit selama lapan minggu berkesan untuk mencegah stres oksidatif pada tikus pos-menopaus.</p>\r\n', 1),
(416, 413, '<p>\r\n	Gamma-tokotrienol (GTT) telah menunjukkan aktiviti antitumor yang signifikan terha&shy;dap pelbagai sel tumor. Penemuan terdahulu menunjukkan bahawa GTT mempunyai kesan antiproliferasi terhadap sel kanser hepar (HepG2) pada nilai IC<sub>50 </sub>170 &mu;M. Di dalam kajian ini, kaedah elektroforesis gel dua dimensi (2DE) digunakan bagi menge&shy;tahui perubahan pada ekspresi protein di dalam sel HepG2 selepas rawatan GTT. Tu&shy;juan utamanya adalah untuk mengenalpasti mekanisme molekul yang mungkin terlibat dalam aktiviti antitumor GTT. Penumpuan diberikan kepada penghasilan profil protein 2DE sel HepG2 dengan dan tanpa rawatan GTT. Analisis awal terhadap profil 2DE, menunjukkan terdapat 18 titik protein diekspreskan secara berlainan di dalam sel di&shy;rawat GTT. Pemerhatian ini dipastikan dengan meluaskan lagi skop kajian kami ke&shy;pada saiz sampel yang lebih besar. Dengan mengkaji kesan rawatan GTT kepada eks&shy;presi protein di dalam sel HepG2, mekanisme asas yang terlibat pada sifat anti tu&shy;mor secara diferensial GTT mungkin boleh dijelaskan.</p>\r\n', 1),
(417, 414, '<p>\r\n	Ekstrak halia telah dilaporkan oleh kumpulan kami sebelum ini yang ianya mempunyai kesan antikanser dan antioksidan dengan menurunkan beban tumor dan peroksidasi lipid pada tikus teraruh kanser. Kajian ini melihat pada pengekspresan protein pro-apoptosis kaspase-8 dan protein anti-apoptosis Bcl-2 pada tikus teraruh kanser. Tiga puluh ekor tikus Wistar dibahagi kepada lima kumpulan ber&shy;dasarkan pada diet yang diberi, iaitu i) kawalan (makanan asas), ii) minyak zaitun iii) ekstrak halia (100 mg/kg berat badan) iv) diet kurang kolin + etionin dalam minuman, CDE (untuk mengaruh kanser) dan v) tikus teraruh kanser, CDE + ekstrak halia. Tikus dibunuh pada minggu ke-lapan dan tisu hepar dikeluarkan untuk pengesanan protein pro-apoptosis dan anti-apoptosis, kaspase-8 dan Bcl-2 melalui kaedah imunohistoki&shy;mia. Pengamatan H&amp;E menujukkan terdapat banyak sel oval yang mengesahkan ter&shy;dapat kanser hepar pada kumpulan yang menerima diet CDE. Terdapat 91.6% (11/12) sampel dari kumpulan CDE yang menunjukkan kehadiran sel oval dan pengeksresan Bcl-2. Tetapi apabila kumpulan CDE dirawat dengan ekstrak halia, sel oval dan peng-ekspresan Bcl-2 me&shy;nurun kepada 8.4% (1/12). Bagi kaspase-8 pula, 41.7% (5/12) sampel dari kumpulan CDE menunjukkan pewarnaan positif dan meningkat ke 100% (12/12) apabila dirawat dengan ekstrak halia. Penemuan dari kajian ini menun&shy;jukkan ekstrak halia mempunyai kesan antikanser dengan mengaruh apoptosis melalui pe&shy;ningkatan protein pro apop&shy;tosis, kaspase-8, dan penurunan protein anti-apoptosis Bcl-2.</p>\r\n', 1),
(418, 415, '<div>\r\n	<p>\r\n		Persekitaran pembelajaran di pusat pengajian tinggi memainkan peranan yang sangat penting dalam menghasilkan graduan yang mempunyai kompetensi yang berkualiti.&nbsp; Didalam program pendidikan kejururawatan, penghasilan graduan yang boleh mem&shy;beri jagarawatan yang holistik kepada komuniti adalah perkara asas yang paling penting. Oleh itu, elemen yang penting adalah aktiviti pengajaran dan interaksi pelajar-pengajar&nbsp; yang berkesan di pusat pengajian tinggi. Untuk mengkaji pengalaman pela&shy;jar kejururawatan UIAM dipelbagai tahun pengajian terhadap pengajar mereka dan menilai hubungkait persepsi pembelajaran persekitaran diantara pengajar dan pelajar. Kajian ini mengunakan kaedah kuantitatif dan menggunakan dua (2) daripada lima (5) bahagian dalam instrumen &lsquo;<em>Dundee Ready Educational Environment Measurement&rsquo;</em> (DREEM).&nbsp; Bahagian yang telah digunakan ialah persepsi pembelajaran oleh pelajar (SPoL) dan persepsi&nbsp; pelajar terhadap pengajar (SPoT) dan jumlah item bagi kedua bahagian tersebut adalah 12 dan 11. Keputusan borang soal selidik menunjukkan pe&shy;lajar kejurururawatan UIAM memberi skor 28.54/48.00 pada (SPoL) dan 28.13/44.00 pada (SPoT). Dapatan kajian kedua bahagian bersetuju pelajar kejururawatan UIAM berpengalaman positif terhadap pengajar mereka dan pembelajaran mereka adalah lebih kearah positif. Keputusan regresi menunjukkan 51% dari jumlah variasi pelajar terhadap persepsi kepada pengajar dijelaskan oleh persepsi pembelajaran oleh pela&shy;jar. Dalam kajian ini jumlah kedua bahagian menunjukkan persamaan dengan kajian terdahalu. Walaupun,&nbsp; keseluruhan skor bahagian (SPoL) dalam kajian ini menuju kategori kearah lebih positif, dua daripada 12 item memperolehi skor yang rendah dari&shy;pada pelajar kejururawatan UIAM. Penyelidik sangat bersetuju dengan mendengar pendapat para pelajar adalah perkara penting untuk sesebuah institusi pendidikan. Oleh itu, pengalihan kepada pembelajaran berpusat pelajar daripada pembelajaran berpusat pengajar haruslah dilaksanakan pada masa kini di institusi-institusi pengajian tinggi. Secara am, keseluruhan min bagi (SPoT) menujukkan para pelajar mempunyai persepsi yang positif terhadap pengajar mereka. Dalam&nbsp; kajian&nbsp; UIAM untuk bahagian ini menunjukkan&nbsp; satu item min skornya adalah rendah daripada 2.00. Dengan itu, kedua bahagian ini menujukkan reflek yang sama iaitu kejayan akademik dan pen&shy;galaman yang positif sebagai pelajar di kampus. Isu yang dikenalpasti dalam kajian di UIAM menunjukkan keperluan untuk mewujudkan suasana yang lebih efektif dengan merancang dan melaksana untuk pembaharuan agar suasana pembelajaran dan pengajaran dapat direalisasikan de&shy;ngan berkesan.</p>\r\n</div>\r\n', 1),
(419, 416, '<p>\r\n	Pendidikan kesihatan merupakan suatu gabungan komponan utama di dalam jaga&shy;rawatan pesakit serangan jantung. Pesakit selepas serangan jantung memerlukan in&shy;formasi berkaitan dengan keadaan semasa mereka supaya dapat mengurangkan rasa gelisah dan membantu dalam proses penyembuhan. Objektif kajian ini adalah untuk mengenalpasti persepsi jururawat terhadap keperluan informasi pesakit pos serangan jantung (myocardial infarction). Kajian rentas yang menggunakan &lsquo;Cardiac Patient Learning Needs Inventory (CPLNI)&rsquo; diadaptasi dari Timmins and Kalizer (2002). CPLNI mengandungi tujuh categori: 1) berkaitan dengan anatomi and fisiologi, 2) faktor psi&shy;kologi, 3) faktor cara hidup, 4) maklumat ubatan, 5) maklumat pemakanan, 6) aktiviti fizikal dan 7) pengurusan gejala. Kajian ini telah dijalankan di unit rawatan jantung &lsquo;CCU&rsquo;, wad pemulihan jantung &lsquo;CRW&rsquo;, wad perubatan 1 dan 2 daripada Januari 2007 ke Mac 2007. 56 responden (96%) yang memenuhi kriteria menyertai kajian ini. Hasil kajian melaporkan jururawat CCU dan CRW mengutamakan pengurusan gejala seba&shy;gai yang pertama diikuti oleh maklumat ubatan, faktor cara hidup, anatomi and fisio&shy;logi, maklumat pemakanan, aktiviti fizikal dan faktor psikologi. Manakala jururawat di&shy;wad perubatan 1 and 2 mengutamakan maklumat ubatan sebagai tahap yang pertama diikuti oleh pengurusan gejala, faktor psikologi, maklumat pemakanan, aktiviti fizikal, anatomi and fisiologi dan faktor cara hidup. Terdapat perbezaan yang signifikan dian&shy;tara persepsi jururawat di wad mereka berkaitan dengan anatomi and fisiologi, faktor psikologi, faktor cara hidup, aktiviti fizikal dan pengurusan gejala dengan nilai p&lt;0.05. Kajian ini merumuskan bahawa keperluan informasi tidak serupa diantara jururawat CCU dan CRW dengan wad perubatan 1 dan 2. Jururawat CCU dan CRW menguta&shy;makan pengurusan gejala manakala jururawat wad perubatan 1 dan 2 mengutamakan maklumat ubatan. Situasi ini perlu diperbaiki agar keperluan informasi pesakit selepas serangan jantung diubahsuai mengikut keperluan setiap individu dan keutamaan bera&shy;saskan keperluan setiap pesakit.</p>\r\n', 1),
(420, 417, '<p>\r\n	Satu kajian keratin rentas telah dilakukan untuk menilai kesesuaian rawatan anemia kekurangan besi semasa dan selepas kehamilan dengan menggunakan suntikan vena <em>iron sucrose complex</em>(Venofer)secara pesakit luar. Seramai 120 pesakit&nbsp; dari Klinik Rawatan Harian Obstetrik, Hospital Universiti Kebangsaan Malaysiatelah dikaji se&shy;lama 18 bulan, diantara March 2003 hingga Ogos 2004. Hasil ukuran utama ialah ke&shy;naikan aras hemoglobin, pematuhan pesakit, kesan sampingan dan kejimatan dari yuran masuk wad. Sebelum menerima rawatan, aras hemoglobin ibu mengandung ialah 8.5<u>+</u>0.85g/dl dan 7.6<u>+</u>0.80 g/dl bagi ibu selepas bersalin. Min kenaikan aras he&shy;moglobin selepas 14 hari merima rawatan ialah 3.52g/dl (2.9- 4.6g/dl).Hasil kajian menunjukkan seorang pesakit mengalami ruam kulit, dan seorang lagi mengalami de&shy;mam panas ringan. Tujuh orang pesakit mengalami rasa metalik. Tiada kesan buruk yang serius atau reaksi anafilaktik berlaku. Sepuluh pesakit (8.3%) tidak meyempurna&shy;kan terapi &ndash; lapan orang ibu bersalin sebelum pelengkapan rawatan, dan dua orang ibu gagal menghadiri diri selepas bersalin. Secara purata, setiap orang pesakit meng&shy;gunakan tujuh ampul Venofer iaitu 700mg, dan menjalani tempoh rawatan selama tiga hari. Rawatan sebagai pesakit luar mengelakkan dari tinggal di wad selama tiga hari dan dapat menjimatkan RM135 untuk rawatan wad kadar RM45 sehari. Dengan itu, sejumlah RM16,200 untuk bayaran masuk hospital dapat dielakkan oleh 120 orang pesakit ini. Sebagai kesimpulan, penggunaan Venofer untuk merawati anemia semasa dan selepas kehamilan sebagai pesakit luar adalah selamat, munasabah, menjimatkan dan mendapat pematuhan pesakit yang tinggi.</p>\r\n', 1),
(421, 418, '<p>\r\n	Jangkitan Methicillin-resistant Staphylococcus aureus (MRSA) kerap berlaku di kalang&shy;an pesakit yang menjalani pembedahan vaskular. Ianya boleh mengakibatkan kehilang&shy;an anggota atau kematian. Kami melaksanakan analisa retrospektif terhadap nota pesakit pembedahan vaskular di Hospital Kuala Lumpur untuk tempoh 2 tahun mulai Januari 2005 hingga Disember 2007. Ianya bertujuan mengamati corak jangkitan dan faktor-faktor yang mengakibatkan jangkitan ini. Seramai 999 pesakit menjalani pembedahan vaskular dalam rangkuman jangkamasa kajian ini. Dari jumlah ini, sera&shy;mai 24 pesakit (2.4%) dikesan terjangkit oleh MRSA. Jangkitan ini didapati terjurus kepada pesakit yang merokok, pesakit diabetes melitus dan mereka yang terdahu&shy;lunya pernah menjalani pembedahan vaskular. Kebanyakan jangkitan bercorak ke&shy;musnahan terhadap luka pembedahan dan berlaku dalam kategori pembedahan ke&shy;cemasan. Sebanyak 54% dari kes jangkitan berakhir dengan kehilangan anggota atau pengeluaran graf vaskular atau kematian. Jangkitan MRSA mengakibatkan kesan klinikal yang buruk terhadap pesakit yang menjalani pembedahan vaskular. Langkah pencegahan yang intensif diperlukan bagi mengelakkan kesan buruk yang tidak di&shy;ingini ini.</p>\r\n', 1),
(422, 419, '<p>\r\n	Dalam kajian ini, pengimejan resonans magnet kefungsian (fMRI) digunakan untuk meng&shy;kaji pengkhususan kefungsian dalam korteks auditori manusia semasa mende&shy;ngar. Para&shy;digma fMRI senyap digunakan untuk mengurangkan artifak bunyi pengim&shy;bas di atas imej kefungsian. Subjek dikehendaki menumpukan perhatian kepada stimulus hingar putih yang diperdengarkan secara binaural pada keamatan 70 dB lebih tinggi daripada aras pendengaran manusia normal. Pengkhususan kefungsian dikaji menggunakan perisian Pemetaan Statistik Berparameter (SPM5) yang berasaskan Matlab melalui analisis kesan malar (FFX), kesan rawak (RFX) dan konjunksi. Analisis individu ke atas semua subjek menunjukkan pengaktifan bilateral yang tidak simetri di antara hemisfera kanan dan kiri pada kawasan Brodmann (BA) 22, 41 dan 42, meli&shy;batkan korteks auditori primer dan se&shy;kunder. Peratus perubahan isyarat didapati lebih besar di ketiga-tiga BA22, 41 dan 42 ka&shy;nan berbanding dengan yang di sebelah kiri (<em>p</em>&gt;0.05). Purata bilangan voksel aktif bagi kesemua kawasan Brodmann tersebut&nbsp; juga didapati lebih tinggi di hemisfera kanan ber&shy;banding dengan kiri (<em>p</em>&gt;0.05). Keputusan FFX menunjukkan titik pengaktifan maksimum berlaku pada BA41 kanan dengan se&shy;banyak 599&plusmn;1 voksel aktif dicerap di lobus temporal kanan berbanding 485&plusmn;1 di lobus temporal kiri. Keputusan FFX didapati tekal dengan keputusan purata bagi kesemua subjek. Analisis konjunksi kemudiannya menunjukkan BA41 kanan dan BA22 kiri se&shy;bagai kawasan pengaktifan sepunya dalam kesemua subjek.&nbsp; Keputusan yang diperolehi mengesahkan pengkhususan korteks auditori kanan dalam pemprosesan stimulus bukan pertuturan.</p>\r\n', 1),
(423, 420, '<p>\r\n	Ini merupakan satu laporan kes klinikal seorang pesakit yang menghidapi menyakit skizofrenia. Biasanya, menggunakan berbagai kombinasi ubat untuk rawatan penyakit ini tidak digalakkan, akan tetapi dengan wujudnya ubat-ubatan baru seperti &ldquo;atypical antipsychotic&rdquo;, cara rawatan sebegini harus dilihat semula. Kes klinikal ini membin&shy;cangkan penggunaan kombinasi ubat-ubatan untuk rawatan pesakit skizofrenia se&shy;belum mereka dimulakan dengan ubat clozapine yang merupakan &ldquo;benteng terakhir rawatan&rdquo;.</p>\r\n', 1),
(424, 421, '<p>\r\n	Kelebihan penyusuan ibu telah pun lama terbukti. Objektif kajian ini adalah untuk mengenalpasti amalan dan pengetahuan penyusuan ibu di dalam&nbsp; komuniti di dalam bandar&nbsp; dan faktor-faktor yang mempengaruhinya. Ini adalah kajian irisan lintang. Ke&shy;semua ibu-ibu yang memenuhi kriteria pemilihan dan menghadiri Klinik Kesihatan Ibu dan Anak di Cheras sepanjang tempoh kajian ini dimasukkan ke dalam kajian. Ibu-ibu tersebut dikehendaki menjawab soal selidik yang diberi. Seramai 162 ibu telah men&shy;gambil bahagian di dalam kajian ini. Didapati prevalen penyusuan ibu dalam minggu pertama selepas bersalin adalah 93.8%. Kajian ini mendapati penyusuan ibu adalah lebih tinggi di kalangan ibu Melayu (<em>p</em>&lt;0.001). Tahap pendidikan ibu, status perkerjaan, perkerjaan suami, pendapatan keluarga dan kelas antenatal tidak mempunyai&nbsp; hubungan yang bermakna dengan amalan penyusuan ibu. Walaubagaimanapun, pengaruh daripada ahli keluarga utama didapati menjadi faktor penting dalam memberi galakan kepada penyusuan ibu. Secara menyeluruh, pengetahuan&nbsp; mengenai penyu&shy;suan ibu adalah baik, tetapi lebih daripada separuh responden (58.6%) tidak tahu ba&shy;hawa mereka perlu meneruskan penyusuan ibu ji&shy;kalau ibu ataupun anak jatuh sakit. Kajian ini menemui bahawa lebih daripada 20% responden masih tidak tahu mengenai &lsquo;lactational amenorrhoea&rsquo; (tiada haid disebab&shy;kan penyusuan ibu), &lsquo;susu ibu&nbsp; melindungi bayi daripada alahan&rsquo; dan &lsquo;luka pada puting adalah satu kebiasaan dalam penyusuan ibu&rsquo;.&nbsp; Lebih kurang 15% daripada ibu-ibu masih tidak tahu bahawa susu ibu adalah lebih baik daripada susu formula. Kesimpul&shy;annya, walaupun prevalen inisiasi penyusuan ibu adalah tinggi di kalangan ibu-ibu, masih terdapat tanggapan-tanggapan yang salah&nbsp; yang perlu diperbetulkan.</p>\r\n', 1),
(425, 422, '<p>\r\n	Penyakit Alzheimer berkait dengan peningkatan kematian sel neuron dan gangguan fungsi kognitif. Penyakit ini dicirikan oleh pembentukan plak di otak. Pengumpulan peptida beta amiloid (A&beta;) dipercayai merupakan penanda awal kepada patofisiologi penyakit Alzheimer. Walau bagaimanapun, mekanisma toksisiti A&beta; masih tidak diketa&shy;hui tetapi mungkin melibatkan peningkatan tekanan oksidatif. Oleh itu, kajian ini dija&shy;lankan untuk menentukan kesan toksik A&beta; terhadap DNA - biomolekul penting yang dioksidakan oleh radikal bebas, dan apoptosis sel serta modulasinya oleh fraksi kaya tokotrienol (TRF), sejenis antioksidan. Sel titian neuroblastoma diperlakukan sama ada dengan 10&mu;M peptida A&szlig;, 5mg/ml TRF diikuti dengan 10&mu;M peptida A&szlig; atau 10&mu;M peptida A&szlig; peptide diikuti dengan&nbsp; 5mg/ml TRF. Sel yang tidak dperlakukan dengan A&szlig; atau TRF dijadikan sebagai kawalan. Kerosakan DNA ditentukan dengan menggu&shy;na&shy;kan asai komet, viabiliti sel ditentukan menggunakan asai 3-(4,5-dimetiltiazol-2-il)-5-(3-karboksimetoksifenil)2-(4-sulfofenil)-2H-tetrazolium (MTS) dan pewarnaan propi&shy;dium iodida dan calcein-AM dijalankan untuk menentukan bilangan sel yang hidup dan sel yang mengalami apoptosis. Keputusan menunjukkan bahawa peptida A&szlig; mening&shy;katkan kerosakan DNA secara signifikan berbanding kawalan (p&lt;0.05) dan mening&shy;katkan kematian sel. Walau bagaimanapun, perlakuan dengan TRF mengurangkan kerosakan DNA secara signifikan, meningkatkan bilangan sel hidup dan mengurang&shy;kan jumlah sel yang mengalami apoptosis berbanding kumpulan yang diaruh oleh peptida A&szlig; sahaja (p&lt;0.05). Oleh itu, kajian ini menujukkan peptida A&szlig; menyebabkan kerosakan DNA dan kematian sel melalui apoptosis kemungkinan disebabkan oleh aruhan kerosakan oksidatif pada DNA. Ini disokong oleh fakta di mana TRF, suatu an&shy;tioksidan, berupaya melindungi kerosakan DNA dan apoptosis.</p>\r\n', 1),
(426, 423, '<p>\r\n	Pemberian ubat merupakan satu aspek dari peranan jururawat dalam bidang kejuru&shy;rawatan. Bersama dengan pelbagai peranan jururawat, mereka juga perlu mempunyai pengetahuan yang mencukupi dan amalan yang baik. Tujuan kajian ini adalah untuk mengetahui tahap pengetahuan, sikap dan amalan jururawat dalam pemberian ubat melalui mulut dan juga untuk menilai samada pengetahuan jururawat, sikap dan ama&shy;lan berkaitan dengan pengalaman bekerja dan pendidikan profesional. Satu kajian rentas telah dijalankan ke atas 40 jururawat yang berkerja di wad medikal, Pusat Peru&shy;batan Universiti Kebangsaan Malaysia. Soal kaji selidik digunakan untuk mengukur tahap pengetahuan dan sikap jururawat dan teknik pemerhatian secara langsung de&shy;ngan menggunakan &ldquo;senarai semak&rdquo; untuk mencatit amalan jururawat. Min skor untuk pengetahuan adalah 13.8, sikap (16.4) dan amalan (10.7) dan berada di antara tabur&shy;an purata. Tiada perbezaan signifikan di antara pengetahuan dan amalan jururawat dengan pengalaman berkerja dan pendidikan profesional.Walaubagaimanapun peng&shy;aruh keatas&nbsp; pengalaman berkerja&nbsp; dan kursus pos basik keatas sikap jururawat dalam pemberian ubat melalui mulut didapati signifikan (p=0.045). Jururawat di wad medikal, Pusat Perubatan Universiti Kebangsaan Malaysia didapati mempunyai tahap pengeta&shy;huan dan sikap yang sederhana dalam pemberian ubat melalui mulut. Kajian ini men&shy;cadangkan pengalaman kerja tidak mempengaruhi pengetahuan dan praktis yang baik dalam pemberian ubat melalui mulut.</p>\r\n', 1),
(427, 424, '<p>\r\n	Melanin merupakan pigmen yang menentukan warna kulit. Sintesis melanin dimang&shy;kinkan oleh enzim tirosinase dan melibatkan pengawalaturan oleh gen <em>TYR, TYRP1 </em>dan<em>TYRP2</em>. Dalam kajian ini, fraksi kaya tokotrienol (TRF) telah digunakan sebagai perencat sintesis melanin. TRF mengandungi 75% alfa-tokotrienol dan 25% tokoferol. Objektif kajian ini adalah untuk menentukan kesan fraksi kaya tokotrienol sawit terha&shy;dap sintesis melanin melalui penentuan kandungan aras melanin, aktiviti enzim tirosi&shy;nase dan ekspresi gen yang mengawalatur sintesis melanin di dalam melanosit kulit iaitu <em>TYR, TYRP1 </em>dan<em>TYRP2</em>. Sintesis melanin ditentukan melalui pengukuran kan&shy;dungan melanin dan asai aktiviti tirosinase. Ekspresi gen diukur dengan menggunakan kaedah kuantitatif PCR transkripsi berbalik (real time RT-PCR). Kultur primer sel mela&shy;nosit kulit dibahagikan kepada dua kumpulan iaitu kumpulan kawalan dan kumpulan perlakuan dengan TRF pada dos 500 &micro;g/ml selama 24 jam. Hasil kajian menunjukkan terdapat penurunan kandungan melanin dan aktiviti tirosinase pada sel melanosit kulit yang diperlakukan dengan TRF berbanding sel kawalan (p&lt;0.05). Ekspresi gen <em>TYRP2</em>menunjukkan penurunan apabila melanosit diperlakukan dengan TRF ber&shy;banding dengan sel kawalan (p&lt;0.05) . Oleh itu, dapat disimpulkan bahawa fraksi kaya tokotrienol sawit bertindak sebagai agen anti pigmentasi semasa penuaan kulit iaitu ia merencat sintesis melanin melalui perencatan aktiviti tirosinase dan menurunkan eks&shy;presi gen <em>TYRP2</em>.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(428, 425, '<p>\r\n	Pesakit semakin berasa tidak puas hati dengan &ldquo;masa menunggu&rdquo; dan &ldquo;tempoh kon&shy;sultasi&rdquo; di klinik pesakit luar. Satu kajian keratan rentas telah dijalankan untuk mengu&shy;kur tempoh menunggu dan tempoh konsultasi pesakit dan mengenalpasti tahap kepu&shy;asan pesakit terhadap tempoh menunggu dan tempoh konsultasi di Klinik&nbsp; Ortopedik, Pusat Perubatan&nbsp; Universiti Kebangsaan Malaysia (PPUKM). Kajian ini turut mengkaji kepuasan pesakit terhadap keseluruhan perkhidmatan klinik. Data dikumpul dengan menggunakan borang soal-selidik isi sendiri. Seramai 81 orang pesakit mengambil bahagian di dalam kajian dengan kadar respons 100 peratus<strong>. </strong>Hasil kajian menunjuk&shy;kan bahawa terdapat perbezaan yang signifikan di antara min tempoh menunggu ta&shy;hap II (tempoh menunggu daripada pesakit diberi nombor sehingga pesakit dipanggil masuk ke dalam bilik doktor) yang dijangka (ekspektasi) dengan min tempoh me&shy;nunggu tahap II sebenar yang dialami. Perbezaan signifikan juga didapati diantara min tempoh konsultasi yang dijangka dengan tempoh konsultasi sebenar. Walau ba&shy;gai&shy;manapun, perbandingan nilai min tempoh menunggu tahap I (tempoh menunggu dari&shy;pada pesakit mendaftar sehingga pesakit diberi nombor) yang dijangka dengan tem&shy;poh menunggu sebenar tidak menunjukkan perbezaan yang signifi&shy;kan (p&gt;0.05). Se&shy;cara keseluruhan, kepuasan pesakit terhadap tempoh menunggu adalah rendah (29.6%) dan tempoh rawatan adalah sederhana (41.9%). Manakala kepuasan keselu&shy;ruhan perkhidmatan klinik menunjukkan tahap kepuasan yang sederhana (56.8%). Kajian ini juga menunjukkan bahawa kepuasan pesakit tidak dipengaruhi oleh faktor sosiodemografi responden seperti umur, bangsa, tahap pendidikan, pekerjaan dan pendapatan. Keputusan penting dari kajian ini akan dapat membantu pihak peng&shy;urus&shy;an klinik dalam masalah aduan pesakit dan mempertingkatkan lagi tahap kepuas&shy;an pesakit terhadap perkhidmatan keseluruhan klinik Ortopedik di PPUKM.</p>\r\n', 1),
(429, 426, '<p>\r\n	Kesakitan selepas pembedahan selalunya dialami oleh pesakit dan menjejaskan per&shy;gerakan serta melambatkan penyembuhan. Kesakitan akut selepas pembe&shy;dahan adalah subjektif dan tidak dapat dinilai secara langsung. Objektif kajian ini ada&shy;lah utuk menilai tahap pengetahuan dan sikap jururawat terhadap pengurusan kesa&shy;kitan sele&shy;pas pembedahan. Kajian berbentuk keratan rentas ini dijalankan di wad sur&shy;gikal dan unit rawatan rapi&nbsp; di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) ber&shy;mula dari Februari hingga April 2008. Terdapat 28 soalan yang mengan&shy;dungi dua kon&shy;struk iaitu pengetahuan dan sikap jururawat terhadap pengurusan kesa&shy;kitan pesakit. Seramai 84 responden menyertai kajian ini. Hasil kajian menunjukkan 21 (25%) ber&shy;pengetahuan tinggi, 58 (69%) responden mempunyai pengetahuan yang sederhana, dan hanya&nbsp; 5 (6%) responden berpengetahuan rendah terhadap pengu&shy;rusan kesakitan selepas pembedahan. Seramai 66 responden (78.5%) menunjukkan sikap positif ter&shy;hadap pengurusan kesakitan selepas pembedahan. Terdapat hubungan yang signifikan di antara sikap positif dan kelulusan akademik jururawat (&chi;&sup2; =29.96, p &lt;0.05). Kesimpulannya kajian ini menunjukkan jururawat di PPUKM mempunyai&nbsp; pengetahuan sederhana dan sikap yang positif terhadap pengurusan kesakitan selepas pembedahan. Hasil kajian ini menunjukkan keperluan&nbsp; pendidikan berterusan dalam pengurusan&nbsp; kesakitan di kalangan para jururawat di PPUKM bagi memperbaiki pengetahuan mereka dalam pengendalian kesakitan demi memperbaiki mutu jagaan selepas pembedahan.</p>\r\n', 1),
(430, 427, '<p>\r\n	Masa menunggu yang lama bagi pembedahan elektif merupakan satu tanda aras perkhidmatan yang tidak efisien. Satu kajian irisan lintang masa menunggu pembeda&shy;han elektif ortopedik dan faktor-faktor yang mempengaruhinya telah dijalankan di se&shy;buah hospital universiti dari Januari 2003 hingga Jun 2004 dengan menggunakan re&shy;kod-rekod &lsquo;case mix&rsquo; yang selesai dikodkan.&nbsp; Hasil kajian utama yang diukur adalah masa menunggu untuk pembedahan elektif dari tarikh temu janji di klinik pakar se&shy;hingga tarikh pembedahan (T1) dan masa menunggu dari tarikh pembedahan diberi&shy;kan sehingga tarikh pembedahan sebenar berlangsung (T2). Median masa menunggu T1 ialah selama 23 minggu (5.75 bulan) dan 5.86 minggu (1.47 bulan) bagi T2. Ujian Khi Kuasa Dua untuk T1 adalah bagi faktor umur dan ko-morbiditi tetapi faktor bangsa hanya bagi T2. Walaubagaimanapun, Ujian Logistik Regresi Berganda mendapati faktor pembayaran pesakit melalui pihak ketiga dengan nilai&nbsp; (OR) 1.97 (95%CI:1.05-3.72) adalah bererti lebih berkemungkinan menunggu lama berbanding pesakit yang membayar sendiri. Bagi T2, wanita dengan nilai&nbsp; (OR) 2.29 (95%CI: 1.19-4.42), India dengan nilai&nbsp; (OR) 2.50 (95%CI: 1.16-5.38) dan bekerja sendiri dengan nilai&nbsp; (OR) 4.28 (95%CI: 1.23-14.97) adalah beerti lebih berkemungkinan mempunyai masa menunggu yang singkat. Oleh itu, faktor penentu bagi keseluruhan masa menunggu ialah umur, ko-morbiditi, bangsa, pekerjaan dan cara pembayaran perkhidmatan oleh pesakit.</p>\r\n', 1),
(431, 428, '<p>\r\n	<em>Staphylococcus epidermidis</em>adalah satu daripada patogen yang semakin penting da&shy;lam hos yang terimunokompromi. Kami melaporkan di sini suatu kes pneumonia berne&shy;krosis akibat <em>S. epidermidis</em> yang dikomplikasi dengan pneumatosel pada seo&shy;rang bayi lelaki yang dilahir cukup bulan. Beliau menghidapi penyakit fistula trakeo-esofa&shy;gus yang perlu dibedah sebelum kejadian ini. Rawatan dengan antibiotik &ldquo;tei&shy;coplanin&rdquo; dan &ldquo;gentamicin&rdquo; diberikan selama empat minggu diikuti dengan &ldquo;erythromy&shy;cin&rdquo; untuk dua minggu. Bayi tersebut tidak perlu menjalani aspirasi perkutaneus atau&shy;pun penyaliran pleura dan ianya pulih sepenuhnya dari segi klinikal dan gambaran ra&shy;diologi apabila ditemu enam bulan kemudiannya semasa lawatan tindaksusulan. Ra&shy;watan dengan ubatan antimikrobial yang agresif mampu menjana pemulihan sepe&shy;nuhnya tanpa mengakibatkan kerumitan dengan komplikasi seperti pneumotoraks dan fistula bron&shy;kopleura.</p>\r\n', 1),
(432, 429, '<p>\r\n	Pengekspresan terlampau P-glikoprotin (P-gp), hasil bagi gen untuk kaset pengikatan ATP (ABC), subfamili B, ahli 1 (ABCB1/MDR1), memain peranan penting dalam mem&shy;perantarai ketahanan <em>multidrug</em> terhadap dadah antiepilepsi (DAE), pada lebih kurang 30% daripada pesakit epilepsi. Variasi genetik mungkin merupakan sebahagian dari&shy;pada sebab kenapa terdapatnya perbezaan inter-individu dalam perhubungan fenotip-genotip bagi respons farmakologikal pesakit epilepsi terhadap DAE. Sinonim poly&shy;morfisme C3435T adalah satu daripada varian alel yang paling kerap berlaku pada gen ABCB1/MDR1, dan ini &nbsp;dicadangkan sebagai penyebab epilepsi refraktori. Banyak kajian telah menunjukkan hubungan antara polimorfisme C3435T dan refraktori terha&shy;dap DAE bagi penyakit epilepsi. Walaupun begitu, terdapat kontroversi antara hasil penemuan dari pelbagai kajian, iaitu, sama ada &nbsp;polimorfisme gen ABCB1/MDR1 C3435T berkait dengan respons terhadap DAE bagi pesakit epilepsi. Kertas ulasan ini memberikan latarbelakang dan membincangkan hasil penyiasatan pada faktor-faktor yang bercanggah yang mempengaruhi interpretasi dan implementasi kajian-kajian asosiasi dalam bidang ini.</p>\r\n', 1),
(433, 430, '<p>\r\n	Pelajar pergigian perlu dilengkapkan dengan kemahiran, ilmu pengetahuan serta sikap positif semasa memberi penjagaan pergigian kepada wargatua. Laporan daripada Kementerian Kesihatan Malaysia menunjukkan prevalens karies pergigian dan penya&shy;kit periodontium di kalangan wargatua negara ini masih tinggi. Tujuan kajian ini adalah untuk mengenalpasti tahap pengetahuan serta persepsi pelajar pergigian tahun akhir terhadap wargatua erkaitan dengan pembelajaran pergigian geriatrik di Univer&shy;siti Ke&shy;bangsaan Malaysia (UKM). Semua pelajar pergigian tahun akhir diminta me&shy;leng&shy;kapkan borang soal selidik yang merangkumi 3 bahagian: 1) <em>Palmore&rsquo;s Facts on Aging Quiz II</em>, 2) Faktor sosial dan kesihatan mulut pesakit, dan 3) Persepsi terhadap pem&shy;belajaran pergigian geriatrik. Sejumlah 83 pelajar telah turut serta dalam kajian ini. Dua puluh empat peratus (n=20) daripada mereka mendapat markah melebihi 70% dalam soalan berkenaan pengetahuan terhadap wargatua, sementara 73% (n=61) mendapat markah di antara 50-70%. Para pelajar telah memilih ketidakupayaan fizikal, masalah pengang&shy;kutan dan keutamaan lain yang lebih kompleks sebagai tiga sebab teratas bagi war&shy;gatua tidak menepati temujanji rawatan pergigian mereka. Majoriti pe&shy;lajar melaporkan kekurangan sumber bahan pendidikan dan pendedahan latihan la&shy;pangan yang terhad sebagai halangan utama yang dihadapi sepanjang kursus. Seba&shy;gai kesimpulan, keba&shy;nyakan pelajar mempunyai tahap pengetahuan yang sederhana berkaitan dengan wargatua serta berupaya mengenalpasti isu sosial yang mempenga&shy;ruhi kesihatan mulut mereka. Secara am, tiada banyak perubahan terhadap persepsi kemahiran pe&shy;lajar pergigian dalam menguruskan pesakit wargatua selepas menjalani modul pergi&shy;gian geriatrik di fakulti.</p>\r\n', 1),
(434, 431, '<p>\r\n	Ini adalah satu kajian retrospektif dengan merujuk kepada rekod-rekod pembedahan dari tahun 2000 hingga 2007. Rekod- rekod pembedahan tersebut diperolehi&nbsp; daripada dua buah pusat rujukan Otorinolaringologi (ORL) Malaysia. Kajian ini bertu&shy;juan untuk menilai struktur demografi, presentasi klinikal,dan rawatan bagi pesakit-pesakit yang mengalami jangkitan abses retrofaring. Terdapat lapan orang pesakit dalam kajian ini, lima perempuan dan tiga lelaki dengan median umur semasa men&shy;dapat rawatan iaitu 47 tahun. Faktor ko-mobiditi di dalam kajian ini adalah diabetis mel&shy;itus, di mana empat orang pesakit menghidap diabetis. Tiga pesakit tertelan tulang ikan. Presentasi yang paling sering dialami oleh pesakit adalah sakit ketika menelan atau kesukaran untuk menelan. Walaubagaimanapun, tiada pesakit yang mengalami demam. Kesemua pe&shy;sakit dirawat dengan antibiotik intravena dan pembedahan. Em&shy;pat pesakit menjalani pembedahan mengeluarkan abses melalui rongga mulut, tiga pesakit menjalani pem&shy;bedahan mengeluarkan abses melalui tepi leher dan abses pe&shy;cah secara spontan terjadi kepada seorang pesakit. Kesemua pesakit sembuh dari jangkitan tersebut ke&shy;cuali seorang sahaja yang meninggal dunia disebabkan septisemia.</p>\r\n', 1),
(435, 432, '<p>\r\n	Penyakit bawaan air yang tercemar (waterborne diseases) adalah masalah berlarutan dan pengancam utama kesihatan ke atas penduduk di kebanyakan negara yang kurang maju disebabkan ketiadaan infrakstuktur asas terutamanya air dan kemudah&shy;an-kemudahan kesihatan. Kajian ini melaporkan hasil penyiasatan penyakit bawaan air yang tercemar di kalangan komuniti yang tinggal di persisiran sungai di <em>Anambra State</em>, di Nigeria. Satu tinjauan dijalankan ke atas 1200 orang penghuni rumah meng&shy;gunakan soal selidik dan 600 rekod yang diambil dari hospital. Fokus utama kajian ini adalah untuk mengenalpasti sosioekonomi dan faktor kebudayaan yang menyumbang kepada epidemiologi penyakit-penyakit ini. Hasil kajian ini menunjukkan perbezaan yang signifikan dalam kejadian penyakit bawaan air yang tercemar di kalangan komu&shy;niti dan di antara komuniti-komuniti lain yang mana mempunyai implikasi tahap pen&shy;didikan, ketua keluarga, pekerjaan dan sumber air yang digunakan. Kajian ini menge&shy;syorkan supaya pendidikan kesihatan komuniti melalui pekerja kesihatan komuniti yang terlatih diberikan dan mewujudkan pusat perkhidmat&shy;an penjagaan kesihatan yang berfungsi, sebagai langkah yang utama untuk menangani penyakit yang merebak melalui penggunaan air yang tercemar.</p>\r\n', 1),
(436, 433, '<p>\r\n	Walaupun stres dikalangankakitangan kejururawatan adalah biasa, mempunyai kae&shy;dah&nbsp; menangani masalah yang efektif dapat membantu meminimakan tahap stres.&nbsp; Objektif kajian ini adalah untuk membandingkan tahap stres di kalangan kakitangan kejururawatan, mengenal-pasti kaedah&nbsp; menangani masalah (coping style)&nbsp; dan me&shy;nentukan hubungan di antara tahap stres dan kaedah&nbsp; menangani masalah . Kajian rentas ini melibatkan 106 kakitangan kejururawatan yang telah disampel&nbsp; secara uni&shy;versal dari wad psikiatri dan jabatan kecemasan di dua buah hospital awam di Lembah Klang. <em>Stress Arousal Checklist</em> (SACL) dan <em>Coping inventory for Stressful Situations</em> (CISS) digunakan untuk menguji tahap stres dan kaedah&nbsp; menangani masalah. Tiada perbezaan signifikan didapati pada tahap stres diantara kedua-dua kumpulan. Dimensi stres pada SACL antara kakitangan psikiatri (6.53 <u>+</u> 3.18, p=0.372) dan kecemasan (6.02 <u>+</u> 2.67, p=0.372) tidak menunjukkan perbezaan signifikan. Dimensi <em>arousal</em> pada SACL antara kakitangan psikiatri (8.60 <u>+</u> 1.70, p=0.372) dan kecemasan (9.19 <u>+</u> 1.61, p=0.07) juga tidak menunjukkan perbezaan signifikan. <em>Task coping</em> adalah kaedah mena&shy;ngani masalah yang paling utama digunakan di kalangan kakitangan psikiatri (55.36 <u>+</u> 9.85) dan kecemasan (57.73 <u>+</u> 9.87). Dimensi stres pada SACL menunjukkan hu&shy;bungan yang signifikan tetapi lemah&nbsp; dengan &lsquo;task coping&rsquo; (r=-0.313,p=0.001) dan &lsquo;emotion coping&rsquo; (r=0.292,p=0.001).Dimensi &lsquo;arousal&rsquo; pada SACL juga menunjukkan hubungan yang signifikan tetapi lemah dengan &lsquo;task coping&rsquo; (r=0.271, p=0.003)&nbsp; dan &lsquo;emotion coping&rsquo; (r=-0.251, p=0.005). Memandangkan profesion kejururawatan mem&shy;punyai tahap stres yang tinggi, hubungan yang signifikan di antara stres dan kaedah&nbsp; menangani masalah membolehkan intervensi dijalankan untuk adaptasi yang lebih baik kepada persekitaran kerja dengan tahap stres yang tinggi.</p>\r\n', 1),
(437, 434, '<p>\r\n	Polip antrokoanal merupakan ketumbuhan benigna yang jarang berlaku. Kami mela&shy;porkan 40 kes polip antrokoanal yang dirawat di Pusat Perubatan UKM selama sepu&shy;luh tahun diantara Mei 1998 hingga April 2008. Median umur pesakit yang terlibat adalah 37 tahun. Simptom klinikal yang sering menjadi masalah utama adalah hidung tersumbat (92.5%), diikuti oleh hidung berair (45%), lelehan belakang hidung (35%) dan berdengkur (22.5%). Median tempoh rawatan susulan adalah 30 bulan. Pelbagai pendekatan pembedahan telah digunakan; pembedahan yang paling kerap dilakukan adalah polipektomi endoskopi dengan peralatan berkuasa elektrik dan antrostomi&nbsp; meatus tengah pada 28 pesakit (70%), diikuti oleh polipektomi endoskopi dan an&shy;trostomi meatus tengah pada tujuh pesakit (17.5%) dengan peralatan konvensional. Lima pesakit (12.5%) menjalani polipektomi endoskopi dengan peralatan berkuasa elektrik tanpa antrostomi meatus tengah. Selain itu, dua pesakit menjalani septoplasti dan seorang pesakit menjalani sinustomi frontal. Terdapat enam pesakit (15%) men&shy;jalani pembedahan kombinasi antrostomi sublabial. Tiada komplikasi besar berlaku ; empat pesakit mengalami penyakit berulang ; tiga pesakit menjalani pembedahan di tempat lain dan seorang daripadanya mengalami penyakit berulang selepas pembe&shy;dahan pertama. Median tempoh penempatan di dalam wad adalah tiga hari. Kami me&shy;rumuskan bahawa penggunaan peralatan berkuasa elektrik dalam rawatan antro&shy;koanal polip adalah selamat, efektif dengan morbidity yang minima.</p>\r\n', 1),
(438, 435, '<p>\r\n	Penambahan kejadian keganasan tempat kerja merupakan masalah yang memerlukan perhatian yang serius yang dihadapi oleh pihak pengurusan di sektor jagaan kesihatan diakhir-akhir ini. Kajian ini telah dilakukan untuk mengenalpasti pengalaman kegana&shy;san tempat kerja yang dialami oleh kakitangan kejururawatan di Pusat Per&shy;ubatan Uni&shy;versiti Kebangsaan Malaysia. Ianya mengenalpasti kategori kejururawatan yang meng&shy;alami kejadian keganasan tempat kerja yang paling tinggi, mereka yang melakukan keganasan dan jenis keganasan yang biasa berlaku. Kajian ini berbentuk keratan rentas yang dijalankan dalam masa tiga bulan iaitu dari bulan Januari hingga Mac, 2004. Jumlah responden, seramai 55 orang, telah menjawab soal selidik dengan pe&shy;nyertaan secara sukarela. Satu set soal selidik telah digunakan untuk mengumpul data berkaitan demografik dan insiden keganasan tempat kerja. Ujian kebolehper&shy;cayaan yang dilakukan menggunakan &lsquo;Crohnbach Alpha&rsquo; menunjukkan 0.872. Kepu&shy;tusan ka&shy;jian menunjukkan kaganasan tempat kerja dikalangan jururawat adalah 3.7 peratus dengan purata 1.2 peratus dalam masa sebulan dan seorang jururawat meng&shy;alami keganasan selang sehari. Jururawat terlatih adalah kumpulan yang paling ramai mengalami keganasan dan mereka yang melakukan keganasan ini adalah pekakit (40.6%) dan diikuti dengan suadara-mara pesakit (37.5%). Jenis keganasan yang paling kerap dilakukan adalah secara &lsquo;verbal&rsquo; (55.6%) yang terdiri daripada penderaan &lsquo;verbal&rsquo; (31.9%) dan ugutan &lsquo;verbal&rsquo; (23.7%). Keputusan ini menunjukkan keganasan tempat kerja adalah satu masalah yang serius dan memerlukan perhatian pihak pen&shy;gurusan. Ini adalah untuk memastikan persekitaran tempat kerja yang selamat bagi para jururawat dan ini akan memastikan penjagaan berkualiti pada pesakit.</p>\r\n', 1),
(439, 436, '<div>\r\n	Sarkoma sel Langerhans adalah proliferasi neoplastik yang jarang ditemui. Ia menunjukan ciri-ciri sitologi yang malignan dan bertindak lebih agresif dari histiositosis sel Langerhans. Dalam&nbsp; laporan ini, kami membentangkan satu kes sarkoma sel Langerhans yang melibatkan vetebra torasiks ke lapan pada seorang lelaki berumur 31 tahun. Walaupun sebelum ini diagnosisnya bergantung pada kehadiran butir Birbeck melalui analisis ultrstruktur, dengan kehadiran antibodi monoklonal yang sesuai pada tisu paraffin, ekspresi CD1a yang khusus pada neoplasma ini dapat ditunjukkan dengan mudah dan seterusnya memberi diagnosis yang spesifik. Dalam laporan ini, kami membincangkan ciri-ciri morfologi dan imunohistokimia dalam diagnosis sarkoma sel Langerhans tanpa analisis ultrastruktur untuk butir Birbeck. Kami juga membincangkan bagaimana immunohistokimia dapat membezakan antara sarkoma sel Langerhans dari neoplasma histiositik dan dendritik yang lain.</div>\r\n', 1),
(440, 437, '<p>\r\n	Seorang pesakit sindrom Rothmund-Thomson (RTS) yang berusia 27 tahun telah di diagnosa dengan kanser sel skuamus dasar lidah. Keab&shy;normalan mekanisma pemulihan DNA pada sebahagian pesakit RTS mendorong mereka untuk mendapat kanser dan keadaan ini juga mempengaruhi toleren kepada rawatan radioterapi yang diberikan.</p>\r\n', 1),
(441, 438, '<p>\r\n	`Metaplastic breast carcinoma&rsquo; (MBC) adalah jenis kanser payu dara yang amat jarang ditemui yang dari segi histologinya, bercirikan karsinoma yang terdiri daripada dua jenis epitelia atau kehadiran karsinoma bersama-sama dengan elemen &lsquo;bukan-epitelial&rsquo;. Ianya ada&shy;lah agresif dan mempunyai ciri prognosis yang buruk. Ciri-ciri &nbsp;patologikal dan klinikal kanser ini dikalangan pesakit kanser payu dara di Pusat Perubatan Uni&shy;versiti Kebangsaan Malaysia (PPUKM) telah dikaji dari tahun 2000 sehingga 2007. Daripada 471 pesakit kanser payu dara di PPUKM terdapat enam&nbsp; pesakit wanita dikenal pasti me&shy;lalui diagnosis histologi mengidap MBC yang mana ianya melibatkan 1.3% dari keselu&shy;ruhan kes kanser payu dara. Empat daripada mereka adalah dari etnik Melayu mana&shy;kala dua adalah India dan min umur mereka adalah 51 tahun. Di dalam kesemua kes, kanser ini adalah besar, bersaiz lebih dari 5 sm, gred 3 dan bereseptor estrogen nega&shy;tif. Lima pesakit menjalani mastectomi (empat bersama &lsquo;axillary clearance&rsquo; manakala satu tanpa &lsquo;axillary clearance&rsquo;). Penglibatan kelenjar limfa aksilari didapati pada empat pesakit (tiga pesakit mempunyai tumor jenis epithelia sahaja dan satu tumor dwifasa). Lima pesakit menerima kemoterapi, seorang lagi enggan dirawat secara kemoterapi. Kesemua pesakit mengalami rekuren tumor dengan min masa re&shy;kuren selama 9 bulan. Kesimpulannya, MBC &nbsp;jarang berlaku, agresif dan biasanya meli&shy;batkan golongan wanita yang sudah menopos. Mereka selalunya bersaiz besar, mem&shy;punyai gred histopatologi tinggi dan bereseptor estrogen negatif. Saiz tumor yang besar serta perebakan tumor ke kelenjar limfa menjangkakan prognosis buruk. Walau&shy;pun dengan rawatan kemoterapi dan radioterapi, pesakit-pesakit ini mempunyai risiko yang tinggi untuk mendapat kanser berulang atau merebak ke organ lain.</p>\r\n', 1),
(442, 439, '<p>\r\n	<em>Chlorella vulgaris, </em>sejenismikroalga unisel, menghasilkan banyak fitokimia intrasel seperti karotenoid, tokoferol, ubikuinon dan protein. Penuaan kulit diaruh oleh tekanan oksidatif melibatkan penurunan sintesis matriks ekstrasel dan peningkatan ekspresi enzim yang mendegradasikan matriks kolagen. Objektif kajian yang dijalankan ini adalah untuk menentukan kesan <em>C. vulgaris </em>terhadap ekspresi gen-gen yang meng&shy;kodkan kolagen (<em>COL</em>) dan matriks metaloproteinase (<em>MMP</em>) yang terlibat di dalam penuaan kulit. Sel fibroblas diploid manusia (HDF) diperolehi daripada kulit khatan kanak-kanak lelaki yang berusia 8-12 tahun. HDF telah dikulturkan kepada 3 kumpul&shy;an: sel kawalan tanpa rawatan, sel dengan penuaan pramatang aruhan tekan&shy;an oksi&shy;datif (SIPS; sel diaruh dengan H<sub>2</sub>O<sub>2</sub> pada pasaj 6 selama 2 minggu) dan SIPS yang dirawat dengan <em>C. vulgaris </em>(rawatan <em>C. vulgaris </em>berpanjangan ber&shy;mula pada pasaj 4 dan gabungan rawatan dengan H<sub>2</sub>O<sub>2</sub> pada pasaj 6 selama 2 minggu). &szlig;-Galaktosidase berkait-penuaan ditentukan menggunakan kit pewarnaan histokimia sel senesen (Sigma, USA). Ekspresi gen <em>COLI, COLIII, COLIV, MMPI, MMPII</em> dan <em>MMPIII </em>dianalisa secara kuantitatif menggunakan kaedah <em>real-time</em> RT-PCR (<em>iScript&trade; One Step real-time PCR with SYBR&reg; Green; </em>Biorad). HDF yang dira&shy;wat dengan H<sub>2</sub>O<sub>2 </sub>(SIPS) me&shy;nunjukkan morfologi sel senesen seperti sel menjadi rata dan membesar dengan pe&shy;ningkatan ekspresi &szlig;-galaktosidase berkait-penuaan (p&lt;0.05). Analisa ekspresi gen menunjukkan <em>COLI</em> menurun di dalam SIPS dan SIPS yang dirawat dengan <em>C. vulgaris </em>(p&lt;0.05)manakala<em>COLIII </em>menurun di dalam SIPS dan meningkat di dalam SIPS yang dirawat dengan <em>C. vulgaris </em>(p&lt;0.05). Ekspresi <em>MMPI</em> meningkat (p&lt;0.05) di dalam SIPS dan SIPS yang dirawat dengan <em>C. vulgaris</em> menunjukkan kesan sinergistiknya bersama dengan rawatan H<sub>2</sub>O<sub>2</sub>. Sebagai kesimpulannya, pada penuaan kulit, ekspresi gen <em>COLI </em>dan <em>COLIII</em> menurun manakala <em>MMPI </em>meningkat. <em>C. vulgaris </em>telah memo&shy;dulasikan ekspresi gen<em>COL </em>dan<em>MMP</em> dengan menurunkan ekspresi <em>COLI </em>dan me&shy;ningkatkan ekspresi <em>COLIII</em> dan <em>MMPI</em> tetapi ia tidak menunjukkan kesan anti pe&shy;nuaan.</p>\r\n', 1),
(443, 440, '<p>\r\n	<em>Helicobacter pylori </em>merupakan agen etiologik gastritis kronik jenis B, ulser peptik dan kanser gaster. Jangkitan <em>H. pylori</em> adalah lazim di dunia dengan anggaran 50% popu&shy;lasi dijangkiti. Majoriti individu yang dijangkiti bersifat asimptomatik, dan sebahagian daripada mereka akan mengalami gastrititis. Walaubagaimanapun, jangkitan kronik <em>H. pylori</em> tanpa rawatan antibiotik mendedahkan individu dijangkiti kepada perkembangan kanser. Tujuan kajian ini adalah untuk menentukan jangkitan aktif <em>H. pylori</em> di kalang&shy;an pesakit dengan simptom dispepsia menggunakan kombinasi 3 kaedah ujian. Da&shy;lam laporan ini, kami mengkaji 1376 pesakit secara konsekutif yang menjalani prose&shy;dur endoskopi di Pusat Perubatan Universiti Kebangsaan Malaysia dari Januari 1999 hingga Disember 2002. Klasifikasi diagnosis pesakit ditentukan melalui pemeriksaan endoskopi dan histologi. Status jangkitan <em>H. pylori</em> ditentukan dengan ujian pantas urease, pemeriksaan histologi atau kultur <em>H. pylori</em>. Jangkitan <em>H. pylori</em> dikesan pada 30.8% pesakit dengan dispepsia. Jangkitan <em>H. pylori</em> ini adalah lebih prevalen pada pesakit dengan usia yang lebih tua dan lebih tinggi pada pesakit lelaki berbanding wa&shy;nita. Pesakit dengan penyakit gastroduodenum parah adalah lebih prevalen dijangkiti <em>H. pylori</em>. Jangkitan <em>H. pylori</em> juga menunjukkan perbezaan yang signifikan di kalangan kumpulan etnik berbeza. Kaum India menunjukkan kadar jangkitan yang paling tinggi iaitu 45.4%, diikuti kaum Cina (36.8%) dan paling rendah pada kaum Melayu (18.3%). Hasil kajian ini yang menentukan jangkitan aktif <em>H. pylori</em> di kalangan pesakit dengan simptom dispepsia adalah selaras dengan kajian serologi terdahulu yang menunjukkan terdapat perbezaan etnik pada prevalen jangkitan <em>H. pylori</em>. Walaubagaimanapun, co&shy;rak jangkitan <em>H. pylori</em> ini tidak selari dengan prevalen penyakit gastroduodenum parah di kalangan kumpulan etnik.</p>\r\n', 1),
(444, 441, '<p>\r\n	Kehadiran sel leukemia yang minima merupakan penanda prognosis buruk pada pe&shy;sakit leukemia akut. Penganalisaan tahap bebas leukaemia secara sitometri aliran da&shy;pat menganggarkan baki minima sel leukaemia (MRD) selepas rawatan kemoterapi induksi pada pesakit kanak-kanak leukemia limfoblastik akut (ALL). Pesakit yang di&shy;katakan bebas dari leukaemia dengan analisa morfologi sum-sum tulang, di mana ka&shy;dar peratus limfoblas adalah kurang 5%, berkemungkinan besar masih mempunyai sel leukemia sebanyak 10<sup>10</sup>.&nbsp; Walau bagaimana pun, kaedah imunofenotip sitometri aliran, boleh mengesan baki sel leukemia yang lebih rendah lagi (1 dalam 10<sup>4</sup> sel-sel) ber&shy;banding pemeriksaan morfologi sumsum tulang, dan ia boleh digunakan sebagai kae&shy;dah bagi menentukan MRD dan meramal kemungkinan leukemia akut berulang.&nbsp; Ka&shy;jian ini adalah untuk membandingkan ciri-ciri klinikal dan hematologi pada kanak-ka&shy;nak yang mengalami ALL, dan juga untuk mengesan status MRD pesakit tersebut se&shy;cara sitometri aliran. Status MRD pesakit dianalisa pada hari ke-28, minggu ke-12 dan minggu ke-20 daripada rawatan kemoterapi induksi leukemia. Jangka hayat pesakit selepas lima tahun juga dikaji. Perbandingan status MRD secara morfologi sum-sum tulang dengan sitometri aliran telah dilakukan. Kajian ini telah dijalankan terhadap tiga puluh lapan kes kanak-kanak yang mengalami prekursor B-ALL di Pusat Perubatan UKM. Tidak terdapat per&shy;kaitan yang signifikan diantara ciri-ciri demografik, klinikal dan hematologi dengan status MRD&nbsp; pada hari ke-28, namun terdapat perkaitan yang sig&shy;nifikan di antara sta&shy;tus MRD yang positif secara sitometri aliran dengan keputusan morfologi sum-sum tulang pada minggu ke-12.&nbsp; Tiga kes menunjukkan status MRD positif yang berterusan sehingga minggu ke-20 dan dua daripadanya telah relaps dan meninggal dunia. Dua puluh empat pesakit masih hidup selepas lima tahun.</p>\r\n', 1),
(445, 442, '<p>\r\n	Neuropati periferi adalah berkait rapat dengan masalah kaki di kalangan penghidap di&shy;abetes melitus. Kajian ini bertujuan untuk mengenalpasti tahap neuropati dan faktor-faktor risiko berkaitan dengan keadaan ini. Satu kajian hirisan lintang telah dijalankan di klinik-klinik ulangan Pusat Perubatan UKM (PPUKM) Malaysia, melibatkan 72 pesa&shy;kit diabetes dan 19 pesakit kawalan.&nbsp; Pesakit yang mempunyai kaki yang bermasalah dan telah mempunyai neuropati tidak dimasukkan dalam kajian. Pesakit kawalan dipi&shy;lih dari mereka yang tidak mempunyai diabetes, boleh berjalan secara normal, tiada masalah dengan kaki serta menghadiri klinik sebagai peneman kepada pesakit. Kajian kuantitatif dilakukan menggunakan Semmes-Weinstein monofilamen. &lsquo;Neuropathy Dis&shy;ability Score&rsquo; (NDS) digunakan untuk mengukur tahap neuropati diabetes. Ujian &lsquo;Spearman rank&rsquo; dan &lsquo;Mann-whitney&rsquo; dijalankan bagi mengenalpasti hubungkait antara neuropati dan faktor-faktor yang dikaji. Ujian &lsquo;logistic regression&rsquo; dijalankan untuk mengenalpasti faktor risiko keadaan ini. Aras purata HbA1c pada pesakit ialah 8.6 <u>+</u> 4.1, dan purata NDS ialah 7.0 <u>+</u> 6.0. Sejumlah 79.1% didapati mempunyai neuropati dalam berbagai tahap, dengan fak&shy;tor kalus kaki dikenalpasti berhubungkait dengan markah NDS yang tinggi. Faktor lanjut usia (p = 0.02), berat badan (p = 0.03), HbA1c (p = 0.005) dan jangkamasa meng&shy;hidap diabetes (p = &lt; 0.005) mempunyai hubungkait positif dengan NDS. Program pen&shy;jagaan kaki yang teratur untuk pesakit diabetes perlu mengambil kira faktor kalus kaki, dengan keutamaan diberi kepada mereka yang mempunyai berat badan berlebihan dan telah lanjut usia.</p>\r\n', 1),
(446, 443, '<p>\r\n	Abses mastoid akut selalu ditemui dalam kerja seharian. Jangkitan ini sering berlaku di kalangan kanak-kanak terutama apabila rawatan yang diberikan tidak berkesan atau rawatan yang diberikan tidak mencukupi untuk menyembuhkan jangkitan tersebut. Be&shy;berapa penulis telah melaporkan bahawa otitis media kronik supuratif mungkin boleh menjadi punca jangkitan tetapi biasanya disebabkan kehadiran &lsquo;cholesteatoma&rsquo;. Di sini, dilaporkan kanak-kanak perempuan berumur 7 tahun yang telah mengalami ab&shy;ses mastoid akut. Kanak-kanak tersebut datang dengan pembengkakan belakang telinga kiri dan kesakitan telinga kiri yang teruk selama 3 hari. Dia juga didapati meng&shy;alami demam. Pemeriksaan telinga kirinya menunjukkan pembengkakan di belakang telinga dan dinding posterior lubang telinganya. Dia telah menjalani pembedahan ke&shy;cemasan mastoid kortikal untuk mengeluarkan nanah dan beliau telah sembuh sepe&shy;nuhnya. Kesimpulannya pesakit ini telah mendapat rawatan antibiotik yang tidak men&shy;cukupi yang meningkatkan risiko beliau untuk menghidapi gejala mastoiditis yang tersembunyi.&nbsp; Ini akhirnya mengakibatkan beliau mendapat abses mastoid. Sehu&shy;bungan dengan itu kami mencadangkan agar semua pesakit yang mengidapi otitis media akut perlulah mendapat rawatan antibiotik selama sekurang-kurangnya se&shy;minggu.</p>\r\n', 1),
(447, 444, '<p>\r\n	Aberasi t(8;21)(q22;q22) kerap berlaku pada leukemia myeloid akut (18-20%) dan se&shy;lalunya dikaitkan dengan sub-jenis &lsquo;French-America-British (FAB) M2&rsquo;. Aberasi trisomi 4 jarang berlaku dan ia adalah keabnormalan spesifik yang berlaku pada AML M2 atau M4. Kami melaporkan satu kajian kes daripada seorang wanita berumur 33 tahun di mana diagnosa klinikal dan laporan hematologi menunjukkan leukemia pro&shy;myelosit akut (APL). Ujian sitogenetik pesakit me&shy;nunjukkan keabnormalan kromosom melibat&shy;kan aberasi t(8;21) dan trisomi kromosom 4. Kehadiran trisomi 4 dengan t(8;21) dalam kes AML jarang berlaku. Kehadiran trisomi 4 dengan t(8;21) tidak mempunyai signifi&shy;kan yang jelas tetapi ia berkait dengan prognosis yang kurang baik.</p>\r\n', 1),
(448, 445, '<p>\r\n	Timoma merangkumi 1% dari keseluruhan kanser mediastinum dan jarang berlaku pada kanak-kanak. Secara tipikal, kanser ini adalah agresif dan kadar sembuh adalah rendah. Rawatan semasa untuk timoma invasif meliputi pelbagai disiplin. Seorang pe&shy;sakit berumur 16 tahun telah di diagnosa dengan kanser timoma in&shy;vasif. Pesakit ini telah berjaya dirawat dengan kimoterapi di ikuti dengan pembedahan dan radioterapi. Setakat ini, pesakit berada dalam keadaan &lsquo;remission&rsquo; yang berterusan selama 6 ta&shy;hun selepas tamat rawatan.</p>\r\n', 1),
(449, 446, '<p>\r\n	Papiloma sinonasal terdiri daripada tiga jenis iaitu &lsquo;inverted&rsquo;, &lsquo;fungiform&rsquo; dan onkositik. Papiloma onkositik merupakan jenis yang jarang berlaku dan ianya lebih kerap terjadi kepada mereka yang telah lanjut usia. Secara klinikal, kelakuannya menyerupai jenis papi&shy;loma &lsquo;inverted&rsquo; kerana mempunyai risiko tinggi mengalami rekuren serta mengan&shy;dungi kom&shy;ponen malignan. Kami melaporkan seorang pesakit yang mempunyai ke&shy;tumbuhan pa&shy;piloma onkositik yang berasal dari sinus maksila sehingga ke kaviti hi&shy;dung dan na&shy;sofarink. Walaupun ketumbuhan ini telah wujud dalam jangkawaktu yang lama tetapi tiada komponen malignan yang ditemui. Pembedahan endoskopi maksilek&shy;tomi medial telah dilakukan untuk membuang ketumbuhan tersebut dengan jayanya. Secara kesimpulannya, diagnosa papiloma onkositik perlu dipertimbangkan pada pe&shy;sakit lanjut usia yang mempunyai polip hidung sebelah. Tambahan pula, pembedahan en&shy;doskopi maksilektomi medial merupakan kaedah pembedahan yang dapat membe-ri&shy;kan keputusan yang memuaskan.</p>\r\n', 1),
(450, 447, '<p>\r\n	Satu kajian irisan lintang telah dijalankan pada tahun 2006 bagi menentukan prevalens infeksi <em>Ascaris lumbricoides</em> di kalangan kanak-kanak Orang Asli di Pos Lenjang, Pa&shy;hang. Sejumlah 71 sampel feses telah diperolehi daripada kanak-kanak (40 perem&shy;puan dan 31 lelaki) yang berumur antara 1-12 tahun. Sampel tersebut kemudiannya diperiksa&nbsp; menggunakan teknik apusan langsung feses dan konsentrasi formalin-eter bagi mengesan kehadiran ova <em>A. lumbricoides</em>.&nbsp; Hasil kajian ini menunjukkan preva&shy;lens infeksi <em>A. lumbricoides</em> secara keseluruhannya adalah 42.3%. Prevalens infeksi <em>Ascaris </em>didapati lebih tinggi di kalangan kanak-kanak lelaki (45.2%) berbanding kanak-kanak perempuan (40%) (p&gt;0.05). Kanak-kanak bersekolah menunjukkan prevalens infeksi lebih tinggi (48.6%) berbanding kanak-kanak pra sekolah (35.3%) (p&lt;0.05). Prevalens infeksi <em>Ascaris lumbricoides</em> yang tinggi di kalangan kanak-kanak&nbsp; Orang Asli ini adalah berkaitan dengan status sosioekonomi, tahap sanitasi persekitaran dan kebersihan diri yang rendah. Daripada 30 sampel yang positif dengan ova <em>A. lumbricoides</em>, didapati teknik konsentrasi formalin-eter menunjukkan peratus penge&shy;sanan 100% berbanding apusan langsung feses dengan peratus pengesanan hanya 46.7% (p&lt;0.05). Oleh itu, adalah disarankan supaya kedua-dua teknik ini (apusan langsung dan konsentrasi formalin-eter) perlu dilakukan dalam pemeriksaan rutin feses untuk diagnosis yang lebih tepat.</p>\r\n', 1),
(451, 448, '<p>\r\n	Satu kajian hirisan lintang telah dilaksanakan bertujuan mengenalpasti prevalens dan distribusi genotip virus papiloma manusia (HPV) di kalangan pesakit prakanser (neoplasia intraepitelial serviks gred 3 atau CIN 3) dan kanser serviks invasif (ICC), di Pusat Perubatan UKM (PPUKM). Blok tisu benaman parafin dari tahun 1999 ke 2007 dikenalpasti dari Jabatan Patologi, PPUKM. Sebanyak 80 rekod perubatan (20 CIN 3&nbsp; dan 60 ICC) dengan informasi lengkap telah dikumpul untuk data klinikopatologi. Dari kanser invasif (n=60), karsinoma sel skuamus (SCC) terdiri dari 75% dan adenokarsinoma 25%. Purata umur pesakit ialah&nbsp; 52.0 &plusmn; 12.21 tahun dan wanita Cina merupakan etnik yang tertinggi (66.3%). Sebanyak 12 jenis genotip HPV telah dikenalpasti iaitu HPV 16, 33, 18, 39, 52, 45, 58, 59, 31, 35, 6 dan 11. Prevalens infeksi HPV ialah setinggi 92.5% dengan genotip 16 yang paling kerap ditemui (73.8%), diikuti dengan genotip 33 (30%) dan genotip 18 (22.5%). Sebanyak 31 kes (38.8%) menunjukkan kehadiran satu jenis genotip HPV sahaja, manakala 44 (53.8%) menunjukkan kehadiran genotip HPV berganda (dua genotip atau lebih). Pada ICC, HPV 16, 33, 18, 52 dan 39 merupakan lima jenis HPV yang paling kerap ditemui. Infeksi HPV yang tinggi dan infeksi berganda merupakan perkara utama ditemui pada lesi CIN 3 dan kanser serviks.</p>\r\n', 1),
(452, 449, '<p>\r\n	Pembedahan pintasan arteri koronari jantung telah menambahkan kadar kemandirian terhadap individu yang menghidapi infarksi miokardium atau iskemia koronari. Kajian ini adalah untuk mengenalpasti persepsi tahap kualiti hidup di kalangan pesakit sele&shy;pas pembedahan pintasan arteri koronari jantung di Institut Jantung Negara. Seramai 69 responden selepas pembedahan pintasan arteri koronari jantung telah menyertai penye&shy;lidikan ini dengan&nbsp; menggunakan borang soal selidik SF 36. Penemuan kajian menun&shy;jukkan responden selepas pembedahan pintasan arteri koronari jantung mem&shy;punyai ta&shy;hap baik kualiti hidup (SF-36) fizikal dan mental. Terdapat perbezaan yang signifikan antara peranan fizikal dalam domain fungsi fizikal kualiti hidup dengan gender respon&shy;den (t=0.286, p&lt;0.05). Tempoh latihan fizikal lebih dari 20 minit mem&shy;punyai kesan positif pada kedua-dua komponen fizikal (t=2,738, p&lt;0.05) dan mental (t=7,326, p&lt;0.05). Pendapatan responden mempengaruhi&nbsp; kedua-dua komponen fi&shy;zikal (F=3.100, p&lt;0.05) dan mental (F=4.272, p&lt;0.05). Kesimpulannya, pesakit sele&shy;pas pembedahan pintasan arteri koronari jantung mempunyai&nbsp; kualiti hidup yang tinggi. Mereka mampu membuat perubahan gaya hidup yang menyeluruh&nbsp; melalui senaman fizikal yang kerap untuk menampung keperluan mereka.</p>\r\n', 1),
(453, 450, '<p>\r\n	Schwannoma saraf kranial ke VII (fasial) merupakan ketumbuhan yang perlahan tum&shy;besarannya dan jarang berlaku, yang terhasil dari sel Schwann pada neurilemma. Ka&shy;jian retrospektif ke atas 6 pesakit yang didiagnosa dengan Schwannoma saraf kranial fasial antara tahun 1998 dan 2008 telah dijalankan. Kajian menunjukkan 3 pesakit adalah perempuan dan selebihnya lelaki. Purata umur mereka adalah 42 tahun (dari 19 hingga 66 tahun). Ketumbuhan tersebut berasal dari bahagian&nbsp; kanal auditori inter&shy;nal (2 pesakit), intra-temporal (3 pesakit) dan intraparotid (1pesakit). Kesemua ketum&shy;buhan tersebut telah dibedah dengan jayanya dan kesinambungan saraf kranial fasial dapat dikekalkan dalam 2 pesakit. Kajian menunjukkan bahawa gejala Schwannoma saraf kranial fasial boleh menyerupai ketumbuhan jenis lain yang melibatkan bahagian saraf kranial fasial yang sama. Pakar bedah mesti sentiasa peka dengan kewujudan&shy;nya terutamanya bila pesakit datang dengan gejala kelemahan saraf fasial yang pro&shy;gresif. Pesakit pula sentiasa perlu diberitahu mengenai kemungkinan komplikasi saraf fasial lumpuh kerana diagnosa Schwannoma saraf kranial fasial hanya dapat dikenal&shy;pasti semasa waktu pembedahan.</p>\r\n', 1),
(454, 451, '<p>\r\n	Satu siri kes retrospektif untuk menyelidik peranan dan keselamatan suntikan pre-operatif faktor <em>anti-vascular endothelial growth</em> (anti-VEGF) ranibizumab &ldquo;LUCENTIS<sup>TM</sup>&rdquo; untuk pesakit yang mengidap penyakit retinopati diabetes yang perlu menjalani pembedahan vitrektomi telah dijalankan. Siri ini melibatkan dua puluh mata konsekutif dari 16 pesakit yang menerima suntikan tersebut 3 hingga 8 hari (purata 4.4 hari) sebelum vitrektomi untuk retinopati diabetis. Umur pesakit adalah dalam lingkungan 46 hingga 72 tahun (purata 57.7 tahun). Tiada berlaku sebarang komplikasi setempat ataupun sistemik. Indikasi untuk vitrektomi adalah <em>retinal detachment</em> (RD) [n=11; 3 kombinasi <em>tractional</em> (TRD) - rhegmatogenous RD (RRD), 8 TRD], TRD dengan pendarahan vitreus (VH) (n=3) ,VH (n=8) dan sindrom traksi vitreo-makular (n=1). Kriteria kemasukan ialah semua kes konsekutif yang menjalani vitrektomi yang ulung-ulung kali menerima suntikan anti-VEGF sebelum pembedahan. Tahap penglihatan (VA) sebelum pembedahan ialah 6/36 hingga persepsi cahaya. Semua mata berdarah dengan kadar minima ke sederhana semasa pembedahan. Pendarahan (VH) selepas pembedahan dalam mata yang tiada tamponad ataupun tamponad gas adalah tiada (n=1), minima (n=13) ataupun sederhana (n=1). Tahap pendarahan pada mata berisi minyak silikon pula adalah tiada (n=1), sederhana (n=3) ataupun pendarahan teruk (n=1). Tahap penglihatan (VA) post-operatif adalah tidak berubah (n=2) (10%), lebih baik (n=14) (70%) ataupun berkurangan (n=4). VA adalah 2/60 atau lebih baik (n=15) hingga tiada persepsi cahaya (n=1). Dua mata mencapai VA sebaik 6/12 (10%). Sepuluh mata (50%) mencapai 6/36 atau lebih baik. Kesimpulannya ialah suntikan pre-operatif intravitreal ranibizumab adalah selamat dan berkesan digunakan untuk pembedahan vitrectomi diabetik dan berupaya mengurangkan pendarahan perioperatif sejurus mampu memulihkan penglihatan pesakit.</p>\r\n', 1),
(455, 452, '<p>\r\n	Kolesteatoma kongenital merupakan satu penyakit yang amat jarang ditemui. Ia dikenali sebagai&nbsp; jisim berwarna putih mutiara di sebalik gegendang telinga yang utuh dan normal yang terdapat pada individu yang tidak pernah menjalani sebarang pembedahan telinga. Kes ini melaporkan kanak-kanak lelaki, berumur 6 tahun dengan ketidakselesaan pada telinga kiri selama setahun, diikuti dengan pernanahan sejak dua bulan sebelum mendapat rawatan. Pemeriksaan telinga menunjukkan gegendang telinga kirinya berlubang di bahagian atik. Pemeriksaan radiologi mendapati kanak-kanak ini menghidapi kolesteatoma peringkat akhir yang memenuhi ruang mastoid, mesotimpanum, dan epitimpanum, merosakkan semua osikel termasuk plat kaki stapes. Mastoidektomi radikal telah dijalankan. Di sini, klasifikasi rawatan dan teori patogenesis kolesteatoma congenital dibincangkan<em>.</em></p>\r\n', 1),
(456, 453, '<p>\r\n	Genom manusia mengandungi banyak variasi genetik submikroskopik dari segi salinan nombor; ini termasuk delesi, duplikasi dan insersi. Walaupun analisis kromosom melalui teknik kariotip masih digunakan sebagai ujian utama dalam penyiasatan pesakit dismorfisma yang juga mengalami masalah terencat akal, teknologi molekular seperti hibridasi genomik perbandingan barisan (<em>array comparative genomic hybridisation</em>, aCGH) terbukti sensitif dan boleh dipercayai dalam mengenalpasti variasi genetik submikroskopik ini. Seorang bayi berumur tiga bulan dirujuk kerana masalah dismorfisma, mikrokefali dan rencatan perkembangan global. Ujian kariotip pertama adalah normal. Walaubagaimanapun, keputusan ini adalah berdasarkan kualiti metafasa yang kurang memuaskan. Ujian aCGH telah mengenalpasti delesi bersaiz 30.6Mb pada kromosom 5p15.33-p13.3. Kes ini menunjukkan keberkesanan aCGH sebagai ujian makmal bagi mengenalpasti masalah ketidakseimbangan kromosom.</p>\r\n', 1),
(457, 454, '<p>\r\n	Limfoma tiroid primer merupakan penyakit kanser yang jarang berlaku. Ia merangkumi 2% daripada keseluruhan limfoma malignan dan kurang daripada 5% daripada lim&shy;foma tiroid. Ia adalah penyakit yang sangat agresif dan merbahaya. Kebanyakan lim&shy;foma ti&shy;roid terdiri daripada limfoma &lsquo;non-Hodgkin&rsquo; yang berasal dari sel limfoid B. Majo&shy;riti kes-kes berlaku dikalangan wanita enam puluhan. Kami melaporkan dua kes lim&shy;foma tiroid primer dengan memberi penekanan terhadap isu-isu klinikal dan kesulitan menangani penyakit ini. Kedua-dua pesakit datang ke hospital dengan kesukaran per&shy;nafasan yang memerlukan pembedahan. Keberkesanan radioterapi, kemoterapi, pem&shy;bedahan dan penggunaan antibodi monoklonal masih lagi didebatkan. Peranan pem&shy;bedahan di dalam menangani kes limfoma tiroid primer sudah banyak berubah namun peranannya yang penting di dalam kes-kes kecemasan tidak boleh diketepikan. Ke&shy;dua-dua pesakit yang dilaporkan perlu menjalani pembedahan kecemasan tetapi hanya seorang sahaja yang telah menerima rawatan tambahan kemoterapi dan ra&shy;dioterapi. Kedua-dua kes ini me&shy;maparkan kesukaran di dalam pengurusan kes lim&shy;foma tiroid primer.</p>\r\n', 1),
(458, 455, '<p>\r\n	Fitosterol&nbsp; adalah&nbsp; sterol&nbsp; tumbuhan&nbsp; yang&nbsp; mempunyai&nbsp; struktur kimia&nbsp; menyerupai&nbsp; koles- terol.&nbsp; Ia&nbsp; mempunyai&nbsp; kesan&nbsp; anti-kolesterol,&nbsp; anti-kanser&nbsp; dan&nbsp; anti-oksida&nbsp; yang&nbsp; mungkin disumbang oleh perencatan peroksidasi lipid. Bagaimanapun, kajian mengenai kesan fitosterol ke atas peroksidasi lipid adalah terhad. Tujuan kajian ialah untuk menentukan kesan&nbsp; fitosterol&nbsp; ke&nbsp; atas&nbsp; malondialdehid&nbsp; (MDA)&nbsp; plasma&nbsp; dan organ&nbsp; pada&nbsp; model&nbsp; tikus yang&nbsp; terdedah&nbsp; kepada&nbsp; karbon&nbsp; tetraklorida.&nbsp; Tikus&nbsp; kajian&nbsp; dibahagikan&nbsp; kepada&nbsp; empat kumpulan iaitu Kawalan normal (NC), Karbon tetraklorida (CCl4), Fitosterol (P) dan Fi- tosterol + karbon tetraklorida (P+CCl4). Kumpulan P dan P+CCl4 menerima pra-rawa- tan&nbsp; fitosterol&nbsp; secara&nbsp; subkutaneous&nbsp; pada dos&nbsp; 140 mg/kg&nbsp; sekali&nbsp; seminggu&nbsp; selama&nbsp; 5 minggu, sementara kumpulan NC and CCl4&nbsp; hanya menerima minyak zaitun (vehikel). Satu dos oral karbon tetraklorida diberikan kepada tikus kumpulan CCl4&nbsp; dan&nbsp; P+CCl4 untuk&nbsp; mengaruh&nbsp; peroksidasi&nbsp; lipid.&nbsp; Selepas&nbsp; 24 jam,&nbsp; kesemua&nbsp; tikus&nbsp; dikorbankan&nbsp; dan MDA&nbsp; plasma&nbsp; dan organ-organ&nbsp; diukur.&nbsp; Seperti&nbsp; jangkaan,&nbsp; karbon&nbsp; tetraklorida&nbsp; telah meningkatkan&nbsp; aras&nbsp; MDA&nbsp; plasma&nbsp; dan hepar&nbsp; kumpulan&nbsp; CCl4&nbsp; berbanding&nbsp; kumpulan kawalan&nbsp; normal.&nbsp; Pra-rawatan&nbsp; fitosterol&nbsp; (kumpulan&nbsp; P+CCl4 )&nbsp; berjaya&nbsp; menghalang peningkatan MDA tersebut. Rawatan fitosterol pada tikus normal&nbsp; (kumpulan P) telah menurunkan&nbsp; aras&nbsp; MDA&nbsp; hepar.&nbsp; Rumusan&nbsp; kajian&nbsp; ialah&nbsp; fitosterol&nbsp; adalah&nbsp; efektif&nbsp; sebagai perencat&nbsp; peroksidasi&nbsp; lipid.&nbsp; Ia berpotensi&nbsp; sebagai&nbsp; suplemen&nbsp; untuk&nbsp; mengurangkan peroksidasi lipid pada individu yang sihat.&nbsp;</p>\r\n', 1),
(459, 456, '<p>\r\n	Kanser&nbsp; payudara&nbsp; adalah&nbsp; kanser&nbsp; yang&nbsp; paling&nbsp; sering&nbsp; ditemui&nbsp; pada&nbsp; wanita&nbsp; di&nbsp; seluruh dunia. Insiden kanser ini semakin meningkat dan merupakan penyebab kematian tertinggi.&nbsp; Kanser&nbsp; payudara&nbsp; diklasifikasikan&nbsp; sebagai&nbsp; sporadik&nbsp; atau&nbsp; famililal&ndash;faktor&nbsp; risiko yang paling kuat adalah sejarah keluarga. Mutasi germline pada gen suseptibel kanser payudara&nbsp; penetran&nbsp; tinggi&nbsp; BRCA&nbsp; 1&nbsp; dan&nbsp; BRCA&nbsp; 2&nbsp; memberi&nbsp; implikasi&nbsp; kuat&nbsp; predisposisi genetik dalam lebih kurang 20% kanser familial. Walaupun BRCA 1 dan BRCA 2 tidak terlibat dalam semua kanser payudara familial, hingga kini, tiada terdapat gen-gen lain yang&nbsp; dikenalpasti&nbsp; mempunyai&nbsp; segregasi&nbsp; kuat&nbsp; dalam&nbsp; kanser&nbsp; payudara&nbsp; familial.&nbsp; Sungguhpun&nbsp; terdapat&nbsp; kajian&nbsp; untuk&nbsp; mengenalpasti&nbsp; faktor-faktor&nbsp; risiko&nbsp; berkaitan&nbsp; kanser payudara,&nbsp; varian-varian&nbsp; yang&nbsp; dikenalpasti&nbsp; melalui&nbsp; kaji&nbsp; asosiasi&nbsp; keluasan-genom (GWAS),&nbsp; hanya&nbsp; menunjukkan&nbsp; peningkatan&nbsp; sederhana&nbsp; risiko&nbsp; kanser&nbsp; payudara&nbsp; dan&nbsp; di masa&nbsp; kini tidak menunjukkan utiliti&nbsp; klinikal. Ulasan&nbsp; ini akan membincang faktor-faktor risiko genetik yang telah diketahui dalam perkembangan kanser payudara dan sejauh mana kemajuan bidang ini semenjak BRCA 1 dikenalpasti.</p>\r\n', 1),
(460, 457, '<p>\r\n	CD151 adalah gen tetraspanin yang terlibat dalam perkembangan dan perebakan sel- sel barah. Ekspresi gen ini meningkat pada kanser payudara gred tinggi, reseptor es- trogen negatif dan c-erbB-2 positif. Walaubagaimanapun, fungsi biologi dan fenotaip di kalangan pesakit&nbsp; yang mempunyai status barah yang berbeza, status reseptor estro- gen&nbsp; (ER),&nbsp; status&nbsp; reseptor&nbsp; progesteron&nbsp; (PR)&nbsp; dan&nbsp; ekspresi&nbsp; c-erbB-2&nbsp; di&nbsp; Malaysia&nbsp; yang terdiri&nbsp; daripada&nbsp; pelbagai&nbsp; kaum,&nbsp; masih&nbsp; belum&nbsp; dikaji&nbsp; secara&nbsp; teliti.&nbsp; Kami&nbsp; menggunakan teknik real-time polymerase chain reaction (qRT-PCR) kuantitatif untuk mengukur eks- presi gen CD151 bagi 45 kanser payudara. Hasil kajian menunjukkan bahawa ekspresi CD151 adalah tinggi pada kanser payudara ER positif (95% interval konfiden) dan PR positif&nbsp; (99%&nbsp; interval&nbsp; konfiden).&nbsp; Sebaliknya,&nbsp; tiada&nbsp; korelasi&nbsp; didapati&nbsp; antara&nbsp; ekpresi CD151&nbsp; dengan&nbsp; gred&nbsp; tumor&nbsp; atau&nbsp; status&nbsp; c-erbB-2&nbsp; pada&nbsp; 95%&nbsp; interval&nbsp; konfiden.&nbsp; Hasil kajian&nbsp; setakat&nbsp; ini mencadangkan&nbsp; bahawa&nbsp; CD151&nbsp; berpotensi&nbsp; sebagai&nbsp; penanda prognostik&nbsp; dan sasaran&nbsp; terapeutik&nbsp; untuk&nbsp; rawatan&nbsp; pesakit&nbsp; payudara&nbsp; estrogen dependen.&nbsp;</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(461, 458, '<p>\r\n	Satu&nbsp; kajian&nbsp; keratan&nbsp; rentas&nbsp; telah&nbsp; dilakukan&nbsp; untuk&nbsp; membandingkan&nbsp; tahap&nbsp; kemampuan pesakit mengingat kembali maklumat lisan dan bertulis tentang rawatan ortodontik. Para peserta merupakan pesakit dan ibu bapa yang menghadiri klinik penyaringan ortodontik, Universiti&nbsp; Kebangsaan&nbsp; Malaysia&nbsp; (UKM).&nbsp; Penyertaan&nbsp; adalah&nbsp; secara&nbsp; sukarela.&nbsp; Peserta dibahagikan kepada dua kumpulan. Kumpulan pertama diberi maklumat bertulis sahaja dalam bentuk risalah, manakala kumpulan yang kedua diberi maklumat lisan sahaja. Isi kandungan maklumat merangkumi 13 item berdasarkan Risalah Maklumat Pesakit yang diterbit&nbsp; oleh&nbsp; The&nbsp; British&nbsp; Orthodontic&nbsp; Society.&nbsp; Kedua-dua&nbsp; kumpulan&nbsp; menerima&nbsp; isi kandungan&nbsp; maklumat&nbsp; yang&nbsp; sama&nbsp; dalam&nbsp; bahasa&nbsp; pilihan&nbsp; mereka.&nbsp; Soal&nbsp; selidik&nbsp; soalan bertutup yang diisi sendiri diberi kepada subjek selepas 15 minit untuk menilai tahap in- gat kembali. Jawapan yang diberi oleh kedua-dua kumpulan peserta dibandingkan. Data yang dikumpul dianalisis dengan SPSS versi 15.0.&nbsp; Ujian chi-square (p=0.05) dijalankan untuk&nbsp; menentukan&nbsp; kesan&nbsp; kaedah&nbsp; pemberian&nbsp; maklumat&nbsp; terhadap&nbsp; peratusan&nbsp; ingat kembali. Seramai 79 subjek menyertai secara sukarela. Terdapat lebih ramai perempuan berbanding&nbsp; lelaki.&nbsp; Majoriti&nbsp; didapati&nbsp; mencapai&nbsp; tahap&nbsp; pengajian&nbsp; sekolah&nbsp; menengah. Perbandingan&nbsp; untuk&nbsp; setiap&nbsp; item&nbsp; soalan&nbsp; mendapati&nbsp; tiada&nbsp; perbezaan&nbsp; signifikan&nbsp; antara mereka&nbsp; yang&nbsp; diberi&nbsp; maklumat&nbsp; lisan&nbsp; atau&nbsp; bertulis.&nbsp; Kedua-dua&nbsp; kumpulan&nbsp; menunjukkan peratusan respons betul yang tinggi. Sebagai kesimpulan, tiada perbezaan dalam tahap ingat kembali pesakit, sama ada pesakit diberi maklumat lisan atau bertulis.</p>\r\n', 1),
(462, 459, '<p>\r\n	Kerentanan terhadap merokok yang ditakrifkan sebagai kekurangan komitmen kognitif untuk&nbsp; tidak&nbsp; merokok&nbsp; di&nbsp; masa&nbsp; depan,&nbsp; telah&nbsp; terbukti&nbsp; sebagai&nbsp; pembolehubah&nbsp; peramal bagi&nbsp; permulaan&nbsp; amalan&nbsp; merokok&nbsp; di&nbsp; kalangan&nbsp; remaja&nbsp; di&nbsp; negara-negara&nbsp; maju. Penyelidikan&nbsp; ini&nbsp; bertujuan&nbsp; untuk&nbsp; menilai&nbsp; kegunaan&nbsp; kerentanan&nbsp; terhadap&nbsp; merokok sebagai pembolehubah peramal bagi permulaan amalan merokok di kalangan remaja di daerah&nbsp; Kota Tinggi, Johor Malaysia. Kerentanan terhadap merokok dinilai dengan menggunakan&nbsp; dua&nbsp; item&nbsp; di&nbsp; kalangan&nbsp;&nbsp;&nbsp; 1763&nbsp; remaja&nbsp;&nbsp;&nbsp; yang&nbsp; tidak&nbsp; merokok&nbsp; pada&nbsp; 2008. Seramai&nbsp; 1288&nbsp; dari&nbsp; 1763&nbsp; remaja&nbsp; (73.1%)&nbsp; yang&nbsp; menyertai&nbsp; kajian&nbsp; memberikan maklumbalas&nbsp; dalam&nbsp; kajian&nbsp; susulan&nbsp; dua&nbsp; belas&nbsp; bulan&nbsp; kemudian.&nbsp; Keputusan&nbsp; kajian menunjukkan 14.9% (n=188/1260) responden adalah rentan terhadap merokok, 31.9% daripadanya&nbsp; mula&nbsp; merokok&nbsp; selepas&nbsp; tempoh&nbsp; setahun.&nbsp; Responden&nbsp; yang&nbsp; rentan terhadap&nbsp; merokok&nbsp; adalah&nbsp; 3.7&nbsp; kali&nbsp; (95%;&nbsp; SK:&nbsp; 2.17-6.30)&nbsp; lebih&nbsp; bermungkinan&nbsp;&nbsp;&nbsp; untuk mula&nbsp; merokok&nbsp; berbanding&nbsp; dengan&nbsp; responden&nbsp; yang&nbsp; tidak&nbsp; rentan&nbsp; terhadap&nbsp; merokok, selepas&nbsp; kesan&nbsp; pemboleh&nbsp; ubah&nbsp; jantina,&nbsp; lokasi&nbsp; sekolah,&nbsp; peratusan&nbsp; rakan-rakan&nbsp; yang merokok,&nbsp; bapa&nbsp; merokok,&nbsp; penerimaan&nbsp; ibubapa&nbsp; terhadap&nbsp; amalan&nbsp; merokok,&nbsp; dan kepercayaan&nbsp; tentang&nbsp; kesan&nbsp; positif&nbsp; dan&nbsp; negatif&nbsp; merokok&nbsp; dikawal.&nbsp; Kajian&nbsp; ini menunjukkan kerentanan terhadap merokok merupakan pembolehubah peramal yang boleh&nbsp; dipercayai&nbsp; dan&nbsp; boleh&nbsp; digunakan&nbsp; sebagai&nbsp; alat&nbsp; saringan&nbsp;&nbsp;&nbsp; untuk&nbsp; mengenalpasti remaja yang berisiko untuk mula merokok.&nbsp;</p>\r\n', 1),
(463, 460, '<p>\r\n	Virus papiloma manusia (HPV) memainkan peranan penting dalam patogenesis kanser servik. Di seluruh dunia, HPV telah dikesan dalam 99.7% kanser servik. Tujuan utama kajian ini ialah untuk membangunkan kaedah SyBrGreen Real-Time PCR (Tindakbalas Berantai Polymerase-Masa Sebenar SyBrGreen) dan seterusnya menggunakan kaedah ini untuk mengenalpasti jangkitan pelbagai bagi dua jenis subtip HPV risiko tinggi. Lima puluh&nbsp; tujuh&nbsp; sampel&nbsp; positif&nbsp; dari&nbsp; ujian&nbsp; TS-PCR&nbsp; (Tindakbalas&nbsp; Berantai&nbsp; Polymerase-Jenis Spesifik) diuji untuk mengesan prevalen genotip HPV risiko tinggi, HPV 16 dan HPV 18. Oleh kerana wujudnya jangkitan pelbagai, keputusan menunjukkan terdapat 67 genom HPV di dalam 57 sampel positif yang dikaji. Genom HPV 16 telah dikesan pada 55/67 (82%) kes, genom HPV 18 pada 8/67 (12%) kes dan 4 kes adalah jangkitan pelbagai. HPV&nbsp; 16 adalah lebih prevalen,&nbsp; diikuti&nbsp; oleh HPV&nbsp; 18. Keputusan&nbsp; menunjukkan&nbsp; graf pencairan DNA bagi HPV 16 adalah pada suhu puncak 80.4&ordm;C dan nilai C (Cycle threshold) bagi produk spesifik HPV 16 adalah pada pusingan ke 20. Manakala graf pencairan DNA bagi HPV 18 adalah pada suhu puncak 79.4&ordm;C dan nilai C&nbsp; bagi produk spesifik HPV 18 adalah pada pusingan ke 22. Sebagai kesimpulan, kaedah Tindakbalas Berantai&nbsp; Polimerase-Masa&nbsp; Sebenar&nbsp; SyBrGreen&nbsp; mempunyai&nbsp; potensi&nbsp; klinikal&nbsp; dalam penyaringan awal, pengesanan dan pengenalpastian jangkitan oleh pelbagai-jenis HPV bagi peringkat-peringkat yang berbeza dalam pesakit-pesakit kanser servik.</p>\r\n', 1),
(464, 461, '<p>\r\n	Pengemparan spesimen darah untuk menghasilkan plasma kurang platelet merupakan aspek penting bagi ujian koagulasi. Pengurangan masa yang diambil untuk mem-proses spesimen tanpa menjejaskan kualiti keputusan ujian yang dihasilkan adalah ideal bagi ujian rutin segera. Pengemparan untuk mendapatkan plasma kurang platelet selama 15 minit pada 3580 rpm secara rutin bagi ujian koagulasi akan menambahkan tempoh masa pengujian tersebut dan tempoh masa pengujian bagi ujian segera. Ka-jian ini dijalankan untuk mengetahui kesan pengurangan masa pengemparan ujian koagulasi bagi memenuhi tuntutan masa ujian segera (30 minit). Ujian prothrombin time (PT), activated partial thromboplastin time (APTT), fibrinogen dan platelet dijalan-kan ke atas 79 sampel darah yang diterima untuk ujian koagulasi rutin, dengan meng-gunakan dua masa pengemparan berbeza untuk setiap sampel iaitu 15 minit pada 3580 rpm dan lima minit pada 4000 rpm. Ujian paired sample t-test menunjukkan ter-dapat perbezaan yang signifikan bagi pengiraan platelet di antara dua kumpulan ter-sebut (p=0.001). Walaubagaimanapun, tidak terdapat perbezaan signifikan bagi ujian APTT normal (p=0.16), APTT tidak normal (p=0.80), PT tidak normal (p=0.43) dan fi-brinogen (p=0.36) bagi kedua-dua kumpulan tersebut. Kesimpulannya, pengemparan yang dijalankan dalam masa yang singkat iaitu lima minit pada 4000 rpm tidak menje-jaskan keputusan ujian koagulasi rutin (PT, APTT dan fibrinogen). Justeru, keputusan kajian ini akan memberi manfaat dalam mengeluarkan keputusan segera, untuk ujian koagulasi khususnya.</p>\r\n', 1),
(465, 462, '<p>\r\n	Injap uretra posterior biasanya dikesan pada kanak-kanak baru lahir. Kami melaporkan seorang pesakit sindrom Down yang didiagnosa dengan injap uretra posterior pada usia 5 tahun. Beliau mendapat komplikasi injap uretra posterior iaitu infeksi berulang organ testis dan epididimis sebagai gejala awal penyakit tersebut. Kes ini akan membincang-kan presentasi luar biasa penyakit injap uretra posterior pada pesakit sindrom Down, cara mendiagnosanya dan komplikasi akibat penyakit tersebut sekiranya tidak dirawat.</p>\r\n', 1),
(466, 463, '<p class="rtejustify">\r\n	Prevalens&nbsp; penyakit&nbsp; diabetes&nbsp; melitus&nbsp; serta&nbsp; kadar&nbsp; jangka&nbsp; hayat&nbsp; seseorang&nbsp; semakin meningkat&nbsp; secara&nbsp; global.&nbsp; Memandangkan&nbsp; penyakit&nbsp; diabetes&nbsp; melitus&nbsp; seringkali&nbsp; di- kaitkan dengan golongan warga emas, adalah amat penting untuk memastikan paras glukos berada pada tahap yang terkawal, agar komplikasi diabetes dapat dikurangkan, dan&nbsp; mempertingkatkan kualiti&nbsp; kehidupan.&nbsp; Kajian&nbsp; hirisan&nbsp; lintang&nbsp; yang&nbsp; dilakukan di ka- langan pesakit diabetes melitus warga emas di daerah Kulim ini bertujuan untuk&nbsp; me- nentukan prevalens paras glukos terkawal&nbsp; dan faktor-faktor yang mempengaruhinya. Sebanyak tiga ratus dua puluh sembilan responden telah dipilih menggunakan kaedah rawak&nbsp; berstratum&nbsp; yang&nbsp; melibatkan&nbsp; kesemua&nbsp; tujuh&nbsp; buah&nbsp; klinik&nbsp; kesihatan&nbsp; di&nbsp; daerah Kulim.&nbsp; Responden&nbsp; ditemubual&nbsp; secara&nbsp; perseorangan&nbsp; dengan&nbsp; menggunakan&nbsp; borang soal&nbsp; selidik&nbsp; berstruktur.&nbsp; Nilai&nbsp; HbA1c&nbsp; 6.5%&nbsp; dan&nbsp; ke&nbsp; bawah&nbsp; dianggap&nbsp; sebagai&nbsp; kawalan glukos&nbsp; yang&nbsp; baik.&nbsp; Prevalens&nbsp; kawalan&nbsp; paras&nbsp; glukos&nbsp; yang baik&nbsp; adalah&nbsp; 22.5%.&nbsp; Faktor yang mempunyai kaitan signifikan dengan kawalan paras glukos yang baik adalah le- laki ((prevalens nisbah ods terlaras=1.75, 95% SK: 1.02-3.00), berumur 70 tahun dan ke atas (prevalens nisbah ods terlaras=2.48, 95% SK: 1.40-4.39) dan tempoh mengi- dap diabetes melitus kurang dari lima tahun (prevalens nisbah ods terlaras=2.10, 95% SK:1.21-3.64).&nbsp; Kesimpulannya,&nbsp; hasil&nbsp; daripada&nbsp; kajian&nbsp; ini&nbsp; menunjukkan&nbsp; bahawa prevalens&nbsp; paras&nbsp; glukos&nbsp; terkawal&nbsp; yang&nbsp; rendah&nbsp; dipengaruhi&nbsp; oleh&nbsp; faktor&nbsp; gender,&nbsp; umur dan jangkamasa mengidap penyakit tersebut.</p>\r\n', 1),
(467, 464, '<p class="rtejustify">\r\n	Stres memberi kesan negatif ke atas kesejahteraan jururawat pelatih dan menghalang pembelajaran atau&nbsp; memberi motivasi dan&nbsp; menggalakkan pembelajaran. Kajian&nbsp; ini mengkaji&nbsp; tentang&nbsp; stres&nbsp; dan&nbsp; faktor-faktor&nbsp; yang&nbsp; mempengaruhi&nbsp; kehidupan&nbsp; harian pelajar-pelajar kejururawatan Diploma dan Sarjanamuda. Seramai 241 pelajar kejururawatan terlibat&nbsp; dalam&nbsp; projek&nbsp; penyelidikan ini.&nbsp; Hasil kajian ini&nbsp; menunjukkan bahawa pelajar-pelajar kejururawatan yang muda (&lt;21 tahun) mengganggap lebih banyak stres daripada pelajar kejururawatan yang lebih senior (&ge;21 tahun). Pelajar- pelajar program Diploma tahun dua menyatakan signifikan stres daripada rakan-rakan mereka (1 dan 3 tahun). Masalah peribadi adalah isu utama yang membimbangkan di kalangan pelajar-pelajar tahun kedua Diploma. Beban kerja pendidikan kejururawatan dan takut gagal kursus kejururawatan adalah tekanan yang utama di kalangan pelajar kejururawatan pertama program kohort Sarjanamuda berbanding senior mereka. Terdapat gabungan kolerasi signifikan yang negatif dan positif diantara akademik dan skor&nbsp; klinikal&nbsp; di&nbsp; kalangan&nbsp; pelajar&nbsp; kejururawatan&nbsp; Diploma&nbsp; dan&nbsp; Sarjanamuda.&nbsp; Stres pelajar kejururawatan tidak dapat mempengaruhi skor GPA mereka. Terdapat kolerasi signifikan yang negatif diantara stres dan skor klinikal di kalangan pelajar kejururawatan&nbsp; Diploma.&nbsp; Terdapat&nbsp; korelasi&nbsp; signifikan&nbsp; yang&nbsp; negatif&nbsp; diantara&nbsp; umur dengan CGPA dikalangan pelajar Sarjanmuda kejururawatan. Pelajar lelaki dan perempuan kejururawatan Diploma dan Sarjanamuda dianggap tidak ada perbezaan dalam tekanan. Hasil kajian ini memberi maklumat penting untuk pendidikan kejururawatan di institut yang dikaji dalam kajian ini, dan mempunyai implikasi bagi penyelidikan masa depan.</p>\r\n', 1),
(468, 465, '<p class="rtejustify">\r\n	Kajian ini bertujuan menentukan prevalens simptom trek urinari bawahan (LUTS) dan tahap kesedaran di klinik pesakit luar lelaki di Pusat Perubatan Universiti Kebangsaan Malaysia. Borang soal selidik yang mengandungi data demografi, soalan berkaitan pengetahuan, sikap dan praktis terhadap penyakit pembesaran kelenjar prostat (BPH) dan skor simptom prostat antarabangsa (IPSS) telah digunakan. Alat pengukuran kelajuan kencing dan imbasan pundi kencing digunakan untuk menggambarkan fungsi sistem urinari dan keterukan BPH. &ldquo;Dipstick&rdquo; air kencing digunakan untuk menentukan kandungan gula, protein dan darah dalam air kencing. Sejumlah 220 pesakit dikaji. Prevalens LUTS yang sederhana dan teruk adalah 42.7%. Simptom paling kerap ialah kerap kencing waktu malam (78.2%), kerap kencing (58.2%) dan ketidakbolehan mengosongkan pundi kencing sepenuhnya (44.6%). Prevalens gula, protein dan darah dalam kencing ialah 23.6%, 11.4% dan 1.8%. Terdapat hubungan yang signifikan antara peningkatan umur dan keterukan LUTS (p=0.005). Daripada 102 pesakit dengan jumlah air kencing melebihi 150ml, terdapat pengurangan yang signifikan dalam kelajuan maksimum (Q<sub>max</sub>) (p=0.039)dan purata kelajuan (Q<sub>ave</sub>) air kencing untuk setiap peningkatan 10 tahun dalam umur (p=0.001). Kebanyakan pesakit (59.5%) pernah dengar tentang BPH. Seramai 78.2% pesakit akan mendapatkan rawatan jika mengidap LUTS sementara 15.9% pula berkata mereka akan mendapatkan rawatan tradisional. Kesimpulannya, prevalens LUTS adalah tinggi dan tahap kesedaran memuaskan.</p>\r\n', 2),
(470, 467, '<p>\r\n	Walaupun&nbsp; bukti&nbsp; menunjukkan&nbsp; rehabilitasi&nbsp; secara&nbsp; tersusun&nbsp; meningkatkan&nbsp; fungsi keseluruhan, kewujudan rehabilitasi secara berterusan dari hospital ke komuniti masih lagi&nbsp; belum&nbsp; direalisasikan.&nbsp; Perkhidmatan&nbsp; ini&nbsp; seharusnya&nbsp; mengambilkira&nbsp; pelbagai&nbsp; jenis ketidakupayaan&nbsp; and&nbsp; keperluan&nbsp; pesakit-pesakit&nbsp; strok&nbsp; itu&nbsp; sendiri.&nbsp; Kajian&nbsp; ini&nbsp; bertujuan untuk&nbsp; mengkaji&nbsp; kesan&nbsp; perkhidmatan&nbsp; pesakit&nbsp; luar&nbsp; rehabilitasi&nbsp; pelbagai&nbsp; displin&nbsp; untuk pesakit&nbsp; strok&nbsp; di&nbsp; dalam&nbsp; komuniti.&nbsp; Pesakit&nbsp; yang&nbsp; telah&nbsp; didiscaj&nbsp; dari&nbsp; Pusat&nbsp; Perubatan Universiti&nbsp; Kebangsaan&nbsp; Malaysia&nbsp; selepas&nbsp; mengalami&nbsp; serangan&nbsp; strok&nbsp; akut&nbsp; dirujuk kepada&nbsp; Klinik&nbsp; Strok&nbsp; Bersepadu&nbsp; Rehabilitasi&nbsp; (KSBR).&nbsp; Pesakit&nbsp; strok&nbsp; diberikan&nbsp; program bersifat&nbsp; individu&nbsp; yang&nbsp; berpandukan&nbsp; masalah&nbsp; pesakit&nbsp; serta&nbsp; menggunakan&nbsp; aktiviti berpandukan&nbsp; perkerjaan&nbsp; bagi&nbsp; mencapai&nbsp; objektif&nbsp; yang&nbsp; ditetapkan.&nbsp; Pesakit&nbsp; dipantau secara berkala samada sehingga cukup 2 tahun didalam program, ataupun sehingga mereka&nbsp; boleh&nbsp; kembali&nbsp; berdikari&nbsp; dikomuniti.&nbsp; Pemantauan&nbsp; di&nbsp; lakukan&nbsp; menggunakan &lsquo;Modified&nbsp; Barthel&nbsp; Index&rsquo;&nbsp; (MBI)&nbsp; dan&nbsp; &lsquo;Berg&nbsp; Balance&nbsp; Scale&rsquo;&nbsp; (BBS).&nbsp; Seramai&nbsp; 68&nbsp; pesakit dirawat&nbsp; di&nbsp; KSBR&nbsp; selama&nbsp; 2&nbsp; tahun,&nbsp; dengan&nbsp; purata&nbsp; umur&nbsp; 62.4&nbsp; tahun&nbsp; (SD&nbsp; 12.4);&nbsp; purata tempoh&nbsp; telah&nbsp; menghidap&nbsp; strok&nbsp; semasa&nbsp; pemeriksaan&nbsp; pertama&nbsp; di&nbsp; KSBR&nbsp; ialah&nbsp; 11.5 bulan&nbsp; (SD&nbsp; 11.9).&nbsp; Majoriti&nbsp; pesakit&nbsp; (67.4%)&nbsp; mendapat&nbsp; samada&nbsp; dua&nbsp; atau&nbsp; tiga&nbsp; jenis intervensi rehabilitasi. Pengukuran MBI dan BBS menunjukkan peningkatan berkesan dalam&nbsp; masa&nbsp; 12&nbsp; bulan&nbsp; (nilai&nbsp; p&nbsp; 0.006&nbsp; dan&nbsp; 0.017).&nbsp; KSBR&nbsp; membuktikan&nbsp; intervensi rehabilitasi&nbsp; secara&nbsp; tersusun&nbsp; berfaedah&nbsp; untuk&nbsp; meningkat&nbsp; fungsi&nbsp; fizikal&nbsp; dan keseimbangan badan pesakit-pesakit strok.</p>\r\n', 1),
(471, 468, '<p>\r\n	Keracunan Amitraz jarang berlaku dan kes ini tidak pernah dilaporkan dalam kajian di Malaysia.&nbsp;&nbsp;&nbsp; Pengambilan&nbsp; bahan&nbsp; ini&nbsp; membawa&nbsp; kepada&nbsp; kesan&nbsp; sampingan&nbsp; yang membahayakan&nbsp; nyawa.&nbsp; Kami&nbsp; melaporkan&nbsp; satu&nbsp; kes&nbsp; keracunan&nbsp; Amitraz&nbsp; yang&nbsp; berlaku pada&nbsp; seorang&nbsp; dewasa&nbsp; berumur&nbsp; 18&nbsp; tahun.&nbsp; Beliau&nbsp; mengalami&nbsp; hipotensi&nbsp; dan memerlukan&nbsp; sokongan&nbsp; pernafasan&nbsp; serta&nbsp; jagaan&nbsp; seharian&nbsp; dalam&nbsp; unit&nbsp; intensif.&nbsp; Beliau telah&nbsp; diberi&nbsp; rawatan&nbsp; berlandaskan&nbsp; simptom&nbsp; di&nbsp; bawah&nbsp; pengawasan&nbsp; rapat&nbsp; dan&nbsp; telah sembuh dengan cepat dan dibenarkan balik selepas 3 hari.</p>\r\n', 1),
(472, 469, '<p class="rtejustify">\r\n	Keputusan ujian jangkamasa yang berpanjangan di dalam ujian &lsquo;activated partial throm-boplastin time&rsquo; (APTT) dan jangka masa normal untuk ujian &lsquo;prothrombin time&rsquo; (PT) adalah kemungkinan disebabkan samada kehadiran kepincangan dalam faktor pembekuan yang melibatkan faktor VIII, IX, XI atau XII, ataupun kehadiran penghalang pembekuan. Ke-mungkinan penghalang pembekuan ini adalah spesifik terhadap sesuatu faktor pembe-kuan atau ia adalah penghalang pembekuan tidak spesifik seperti &lsquo;lupus anticoagulant&rsquo; yang memberi implikasi terapeutik yang tidak sehaluan. Kami melaporkan satu pesakit yang telah dirujuk kepada kami untuk rawatan dimana pendiagnosanya daripada hospital terdahulu melaporkan ia mempunyai penghalang faktor pembekuan IX dengan keabnor-malan jangkamasa yang berpanjangan di dalam ujian APTT. Hasil lanjutan siasatan mak-mal kami telah menunjukkan pesakit ini mempunyai penghalang pembekuan &lsquo;lupus anti-coagulant&rsquo; melalui ujian faktor &lsquo;phospholipid-dependent&rsquo;. Ujian pencairan, ujian kromoge-nik dan neutralisasi fosfolipid dapat membezakan penghalang pembekuan &lsquo;lupus anticoa-gulant&rsquo; dengan yang faktor pembekuan yang lain. Interpretasi terhadap ujian faktor dengan kehadiran penghalang pembekuan &lsquo;lupus anticoagulant&rsquo; perlu dilakukan dengan berhati-hati untuk mengelakkan diagnosis yang tidak tepat dan pemberian rawatan yang tidak se-suai.</p>\r\n', 1),
(475, 472, NULL, NULL),
(476, 473, NULL, NULL),
(764, 761, 'Penuaan disebabkan oleh akumulasi kerosakan sel akibat tekanan oksidatif. Namun, kebolehsesuaian terhadap beberapa jenis senaman yang dilatih secara kerap telah menghasilkan keputusan memberangsangkan pada peringkat fisiologi dan sel yang ditentukan di kalangan warga tua. Dalam kajian ini, modulasi lemak tubuh, indeks jisim  tubuh,  kadar  denyutan  jantung,  tekanan  darah  dan  profil  biokimia  darah oleh senaman Tai Chi telah ditentukan di kalangan wanita pramenopos berumur 45  ke  atas.  Mereka  menjalankan  senaman  Tai  Chi  dengan  diketuai  seorang pelatih  sebanyak  dua  kali  seminggu  selama  satu  jam  bagi  setiap  sesi.  Parameter dibandingkan dengan data asas selepas 6 bulan. Peratus lemak dan kadar denyutan jantung menurun manakala lipoprotein kepadatan tinggi, jumlah protein, albumin dan kalsium meningkat dengan signifikan. Oleh kerana subjek sedentari kawalan tidak  menunjukkan  perubahan,  kami  menyimpulkan  bahawa  senaman  Tai  Chi yang kerap memperbaiki status kesihatan wanita pramenopos.', 1),
(766, 761, '<p>\r\n	Penuaan disebabkan oleh akumulasi kerosakan sel akibat tekanan oksidatif. Namun, kebolehsesuaian terhadap beberapa jenis senaman yang dilatih secara kerap telah menghasilkan keputusan memberangsangkan pada peringkat fisiologi dan sel yang ditentukan di kalangan warga tua. Dalam kajian ini, modulasi lemak tubuh, indeks jisim tubuh, kadar denyutan jantung, tekanan darah dan profil biokimia darah oleh senaman Tai Chi telah ditentukan di kalangan wanita pramenopos berumur 45 ke atas. Mereka menjalankan senaman Tai Chi dengan diketuai seorang pelatih sebanyak dua kali seminggu selama satu jam bagi setiap sesi. Parameter dibandingkan dengan data asas selepas 6 bulan. Peratus lemak dan kadar denyutan jantung menurun manakala lipoprotein kepadatan tinggi, jumlah protein, albumin dan kalsium meningkat dengan signifikan. Oleh kerana subjek sedentari kawalan tidak menunjukkan perubahan, kami menyimpulkan bahawa senaman Tai Chi yang kerap memperbaiki status kesihatan wanita pramenopos.</p>\r\n', 1),
(920, 915, '<p>\r\n	Pendarahan tidak terkawal yang disebabkan kepatahan tulang pelvis menyumbang kepada morbiditi dan mortaliti berkaitan trauma. Tiga strategi utama yang telah digariskan untuk merawat keadaan ini adalah mengurangkan isipadu ruang pelvis dengan mewujudkan kesan tamponade, menghentikan pendarahan melalui kaedah angioembolisasi keatas salur darah besar, dan penstabilan tulang pelvis melalui fiksator luaran. Ini haruslah dimulakan seawal mungkin. Alatan prahospital yang mampu membenarkan kaedah diatas dilaksanakan akan banyak membantu didalam perawatan pesakit yang mengalami keadaan ini. Buat masa ini kebanyakan alatan atau kaedah yang digunakan di prahospital mempunyai keistimewaan mereka tersendiri namun tidak kurang juga terdapat beberapa kekangan. Bagi mengatasi masalah ini, satu senarai semak yang mengandungi ciri-ciri alatan yang sempurna telah dibangunkan. Sepasukan penyelidik dari Universiti Kebangsaan Malaysia (UKM) telah menyahut cabaran untuk merangka dan membina alatan perubatan berpandukan ciri-ciri tersebut. Projek dua fasa selama tujuh tahun telah dilaksanakan melibatkan kajian anthropometrik, biomekanikal, kadaver dan radiologikal dalam menghasilkan alatan yang diperlukan. Akhirnya BRIMTM Immobilizer, sebuah alatan perubatan yang mengimobilisasi pelvis dan anggota bawah terhasil. Ianya satu alat yang mesra pengguna, tahan lasak, kos efektif, lut sinar-x, ringan, dan boleh diguna-semula yang menjawab hampir seluruh persoalan alatan yang diperlukan untuk rawatan.</p>\r\n', 1),
(908, 903, '<p>\r\n	Aneusomi merupakan perubahan genetik awal dan cirri-ciri pencirian dalam kebanyakan tumor pejal. Penemuan aneusomi biasanya dikaitkan dengan pesakit kanser dengan prognosis buruk. Penglibatan penyusunan semula gen PAX8-PPAR&gamma; dalam tumorigenesis lesi tiroid folikular telah dikaji. Namun begitu, tidak banyak laporan yang melaporkan kehadiran aneusomi gen PPAR&gamma; pada lokus 3p25 pada lesi tiroid folikular. Penglibatan keabnormalan ini dalam peningkatan diagnosis, pengkelasan atau ramalan keputusan diagnosis masih tidak dapat ditentukan. Kajian ini melaporkan penemuan aneusomi dalam tiga lesi tiroid folikular [dua karsinoma tiroid folikular (FTC) dan satu kes adenoma sel Hurthle (HCA)] yang menunjukkan kehadiran aneusomi 3p25 menerusi teknik penghibridan in-situ berpendaflur (FISH). Trisomi 3p25 telah dicerap dalam satu FTC dan satu HCA manakala satu kes FTC menunjukkan tetrasomi 3p25. Ketiga-tiga lesi tiroid yang dikaji tidak menunjukkan pengekspresan berlebihan protein PPAR&gamma;. Lesi sel Hurthle berbeza dari segi klinikal dan histologikal daripada neoplasma folikular yang lain. Namun, penemuan aneusomi dalam HCA dan FTC menunjukkan kewujudan pertalian biologi yang berterusan di antara neoplasma sel Hurthle dan folikular. Di samping berkongsi ciri-ciri histologi dengan neoplasma tiroid konvensional, neoplasma sel Hurthle mungkin berkongsi perubahan genetik yang sama di peringkat awal pembentukan tumor folikular.</p>\r\n', 1),
(956, 951, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Ascariasis adalah salah satu jangkitan cancing di Malaysia, lebih-lebih lagi di kawasan pendalaman. Kebiasannnya ascariasis lumbricoides yang masuk dan tinggal di dalam salur pencernaan manusia tidak akan menimbulkan apa-apa simptom. Sekiranya cacing itu masuk kedalam saluran empedu, ia akan menyebabkan komplikasi yang dinamakan biliary ascariasis. Satu kes biliary ascariasis yang menunjukan simptom-simptom seperti biliary colic akan dibincangkan. Pesakit ini telah datang ke Hospital Daerah Jelebu mengadu sakit pada bahagian atas dan kanan perut selama dua hari. Siasatan awal daripada sejarah, pemeriksaan dan ujian makmal menunjukkan pesakit inin mempunyai simptom-simptom seperti acut cholecystitis dan pesakit telah ditahan wad dan dirawat dengan antibiotik. Disebabkan simptom yang bertambah buruk, pesakit ini dirujuk ke Hospital Besar yang ber dekataan. Pesakit ini telah menderita dari penyakit infeksi cacing dibahagian empedu setelah di dikonfirmasikan oleh ultrasound HBS, CT-Scan Abdomen dan ERCP, dimana cacing tersebut dikeluarkan melalui procedur endoscopy. Setelah semua pemeriksaan dan procedur, pesakit telah sembuh dan dibenarkan keluar dari wad.</p>\r\n', 1),
(912, 907, NULL, NULL),
(905, 900, '<p>\r\n	Penyebab utama hipertirodisma adalah Penyakit Grave (GD). Ia merupakan penyakit autoimun dimana berlaku pengikatan antibodi terhadap reseptor hormon perangsang thyroid (TRAb). Kajian ini adalah untuk malakukan validasi keatas pengujian TRAb dari segi kejituan, sensitiviti, spesifisiti dan cut-off value. Ujian kejituan (CV) dilakukan secara dalam masa dan sela masa menggunakan dua tahap kawalan kualiti pada julat kepekatan rendah 3.78-7.02 IU/L dan julat kepekatan tinggi 13.5-21.2 IU/L. Untuk menentukan sensitivity, spesifisiti dan nilai cut-off, 124 sampel serum dari 46 GD , tujuh Hashimoto thyroiditis (HD), 11 non autoimmune nodular goitre (NAG), dua kanser tiroid and 58 normal sebagai kawalan telah diambil secara retrospektif. Kejituan dalam masa adalah 2.4% pada kepekatan 3.90 IU/L(julat: 3.78-7.02 IU/l) and 0.8% pada kepekatan 20.80 IU/L (julat:13.5-21.2 IU/l) Kejituan keseluruhan adalah 3.8% pada kepekatan of 3.80 IU/L (julat:13.5-21.2 IU/l) and 1.0% pada 20.8 IU/L (julat:13.5-21.2 IU/l). Analisa Receiver-operating characteristic (ROC) menunjukkan sensitiviti terbaik (94%) and specifisiti terbaik (98%) adalah pada nilai cut-off 1.69 IU/L. Positive predictive value (PPV) dan negative predictive value (NPV) berada pada 95% dan 94%. Pada nilai 1.69 IU/l ini, didapati sensitiviti untuk 29 sampel pesakit yang baru didiagnosa dengan GD berada pada 94%. Dari kajian ini didapati esai TRAb yang mengambil masa 27 minit ini mempunyai kejituan yang baik, sensitivity yang tinggi untuk mengesan penyakit GD dan spesifisiti yang bagus untuk membezakan GD dari penyakit tiroid yang lain dan membantu dalam perawatan pesakit tiroid.</p>\r\n', 1),
(933, 928, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Penyebab utama hipertiroidisme adalah Penyakit Grave (GD). Ia merupakan penyakit autoimun dimana berlaku pengikatan antibodi terhadap reseptor hormon perangsang tiroid (TRAb). Tujuan kajian ini adalah untuk memvalidasi pengujian TRAb dari segi kejituan, sensitiviti, spesifisiti dan nilai cut-off. Ujian kejituan (CV) dilakukan secara dalam masa dan sela masa menggunakan dua tahap kawalan kualiti pada julat kepekatan rendah 3.78-7.02 IU/L dan julat kepekatan tinggi 13.5-21.2 IU/L. Untuk menentukan sensitiviti, spesifisiti dan nilai cut-off, 124 sampel serum dari 46 GD , tujuh tiroiditis Hashimoto (HD), 11 goiter nodular non-autoimmune (NAG), dua kanser tiroid and 58 normal sebagai kawalan telah diambil secara retrospektif. Kejituan dalam masa adalah 2.4% pada kepekatan 3.90 IU/L(julat: 3.78-7.02 IU/l) and 0.8% pada kepekatan 20.80 IU/L (julat:13.5-21.2 IU/l). Kejituan keseluruhan adalah 3.8% pada kepekatan 3.80 IU/L (julat:13.5-21.2 IU/l) dan 1.0% pada 20.8 IU/L (julat:13.5-21.2 IU/l). Analisa Receiver-operating characteristic (ROC) menunjukkan sensitiviti terbaik (94%) dan spesifisiti terbaik (98%) adalah pada nilai cut-off 1.69 IU/L. Positive predictive value (PPV) dan negative predictive value (NPV) berada pada 95% dan 94%. Pada nilai 1.69 IU/l ini, didapati sensitiviti untuk 29 sampel pesakit yang baru didiagnosa dengan GD berada pada 94%. Dari kajian ini didapati esai TRAb yang mengambil masa 27 minit ini mempunyai kejituan yang baik, sensitiviti yang tinggi untuk mengesan penyakit GD dan spesifisiti yang bagus untuk membezakan GD dari penyakit tiroid yang lain dan membantu dalam rawatan pesakit tiroid.</span></p>\r\n', 1),
(936, 931, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Gen penindas p53 dan proto-onkogen Bcl-2 mengkodkan fosfoprotin nuklear dan protin membran mitokondria yang terlibat dalam pelbagai fungsi sel. Kedua-dua protin mempunyai hubung kait dengan tapak jalan kematian sel berprogram disamping memberi informasi prognostik karsinoma payudara. Objektif kajian ini adalah untuk menentukan kaitan onko-protin p53 dan Bcl-2 pada karsinoma payudara dan hubungannya dengan umur pesakit, saiz tumor, peringkat kanser dan gred histologi. Lima puluh sembilan kes kanser payudara dari Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) telah dikaji menggunakan teknik imunohistokimia. Keputusan menunjukkan 45.8% (27/59) kanser payudara adalah imunopositif untuk p53 dan 40.7% (24/59) adalah imunopositif untuk Bcl-2. Terdapat korelasi signifikan di antara ekspresi Bcl-2 dengan peringkat awal kanser (p=0.01). Variabel lain tidak menunjukkan kaitan signifikan. Kaitan songsang didapati di antara p53 dan Bcl-2 (p=0.001). Hasil keputusan mencadangkan regulasi menurun Bcl-2 oleh p53 dalam karsinogenesis kanser payudara.</span></p>\r\n', 1),
(929, 924, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Kepuasan pesakit memainkan peranan yang penting dalam memastikan penggunaan perkhidmatan jagaan kesihatan, rawatan berterusan dan kepatuhan terhadap rawatan. Maka, kajian ini bertujuan untuk menentukan tahap kepuasan pesakit dengan perkhidmatan yang diberikan oleh klinik jagaan primer PPUKM. Satu kajian deskriptif keratan rentas yang melibatkan 317 pesakit yang hadir ke klinik tersebut telah dijalankan daripada Febuari sehingga Mac 2011. Mereka telah dipilih melalui pensampelan rawak secara sistematik. Dengan menggunakan satu borang soal-selidik kepuasan pesakit-46 (PSQ-46) yang telah disahkan di dalam versi Inggeris dan Melayu, kepuasan keseluruhan dan kepuasan dengan lima sub skala (doktor, jururawat, aksesibiliti, kemudahan dan temujanji) diperiksa. Sebahagian besar pesakit (93.1%) secara umumnya berpuas hati dengan perkhidmatan keseluruhan. Di antara lima skala sub, pesakit sangat berpuas hati dengan doktor (96.5%), tetapi hanya 35.6% pesakit berpuas hati dengan kemudahan. Jumlah pesakit yang berpuas hati dengan sub skala yang lain adalah: 86.1% (temujanji), 82.0% (jururawat) dan 68.1% (aksesibiliti). Terdapat perkaitan yang ketara di antara setiap skala sub dan kepuasan keseluruhan (p&lt;0.001 untuk doktor, jururawat, aksesibiliti dan temujanji; p=0.014 untuk kemudahan). Walaubagaimanapun, perkaitan sedemikian tidak kelihatan di antara faktor-faktor sosio-demografi dan kepuasan keseluruhan (p=0.200 untuk jantina, p=0.108 untuk umur, p=0.894 untuk kaum, p=0.146 untuk tahap pendidikan, p=0.653 untuk status pekerjaan, p=0.867 untuk jenis pekerjaan, dan p=0.623 untuk pendapatan keluarga). Kesimpulannya, kebanyakan pesakit merasakan kemudahan klinik adalah substandard tetapi mereka mempunyai tahap kepuasan keseluruhan yang tinggi. Namun, strategi untuk memperbaiki kemudahan masih diperlukan kerana ia dapat meningkatkan lagi kualiti rawatan dan kepuasan pesakit.</span></p>\r\n', 1),
(944, 939, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Aloimunisasi sel darah merah ditakrifkan sebagai pembentukan antibodi di dalam respons kepada antigen asing sel merah melalui proses transfusi atau mengandung. Di kalangan wanita hamil yang tidak mempunyai sejarah transfusi darah, mereka boleh mengalami aloimunisasi pada kehamilan yang lalu atau semasa disebabkan oleh kehadiran antigen sel merah bapa yang diwarisi kepada janin. Kajian ini bertujuan untuk menentukan prevalens aloimunisasi sel darah merah di kalangan wanita hamil tanpa sejarah transfusi darah di PPUKM dan juga untuk mengaitkan kelazimannya dengan nombor kehamilan dan sejarah komplikasi obstetrik. Ini adalah satu kajian keratan rentas di mana 150 wanita hamil telah dipilih secara rawak di klinik antenatal PPUKM. Sepuluh ml darah periferal telah diperolehi selepas persetujuan diambil dari setiap pesakit. Ujian yang dijalankan ke atas sampel-sampel darah tersebut termasuk ujian jenis kumpulan darah ABO dan Rh D dan saringan antibodi dengan menggunakan teknik ujian antiglobulin tidak langsung. Sampel positif terus dikenalpasti untuk pengkhususan antibodi. Dalam kajian kami, majoriti (37.3%) daripada wanita adalah primigravida. Aloantibodi sel darah merah telah dikesan pada dua daripada 150 (1.3%) pesakit yang kemudiannya dikenalpasti sebagai anti-C dan anti-D. Namun tiada seorang pun daripada primigravida yang mengalami aloimunisasi. Seorang wanita dari gravida 2 (2.9%) dan gravida 3 (3.6%) didapati positif bagi aloimunisasi. Salah seorang daripada mereka juga mempunyai sejarah obstetrik buruk. Kajian ini menunjukkan bahawa kelaziman aloimunisasi sel darah merah di kalangan wanita hamil adalah rendah di pusat ini. Walau bagaimanapun, ujian saringan bagi aloantibodi sel darah merah perlu disediakan untuk mengurangkan komplikasi kepada janin atau ibu yang mungkin timbul akibat aloimunisasi sel darah merah.</span></p>\r\n', 1),
(946, 941, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Demam denggi adalah demam yang disebabkan oleh jangkitan virus dan insidens untuk demam denggi bertambah setiap tahun. Manifestasi okular daripada demam denggi adalah pelbagai, termasuk makulopati, pendarahan retina, &lsquo;retinal lesions&rsquo; dan vaskulitis. Penurunan tahap penglihatan dan gangguan penglihatan seperti metamorfopsia adalah presentasi biasa dalam makulopati yang disebabkan oleh demam denggi. Kami melaporkan satu kes yang menarik melibatkan seorang wanita muda dengan makulopati di kedua- dua belah mata disebabkan oleh demam denggi dan diberikan rawatan konservatif. Kisah ini menonjolkan kemungkinan penglibatan okular secara serentak di kedua-dua belah mata di kalangan pesakit yang dijangkiti demam denggi. Doktor perubatan perlu mengambil tahu mengenai komplikasi okular yang disebabkan oleh demam denggi dan menasihatkan pesakit mengenai tanda- tanda yang mungkin berlaku.</span></p>\r\n', 1),
(950, 945, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Aneusomi merupakan perubahan genetik awal dan suatu ciri utama dalam kebanyakan tumor pejal. Penemuan aneusomi biasanya dikaitkan dengan pesakit kanser dengan prognosis buruk. Penglibatan penyusunan semula gen PAX8-PPAR&gamma; dalam tumorigenesis lesi tiroid folikular telah dikaji. Namun begitu, tidak banyak laporan yang melaporkan kehadiran aneusomi gen PPAR&gamma; pada lokus 3p25 pada lesi tiroid folikular. Samada kehadiran keabnormalan ini dapat meningkatkan diagnosis, pengkelasan atau prognosis masih tidak dapat ditentukan. Kajian ini melaporkan penemuan aneusomi dalam tiga lesi tiroid folikular [dua karsinoma tiroid folikular (FTC) dan satu kes adenoma sel Hurthle (HCA)] yang menunjukkan kehadiran aneusomi 3p25 menerusi teknik penghibridan in-situ berpendaflur (FISH). Trisomi 3p25 telah ditemui pada satu kes FTC dan satu kes HCA manakala satu kes FTC menunjukkan tetrasomi 3p25. Lesi sel Hurthle berbeza dari segi klinikal dan histologikal daripada neoplasma folikular yang lain. Namun, penemuan aneusomi dalam HCA dan FTC menunjukkan kewujudan pertalian biologi di antara neoplasma sel Hurthle dan folikular. Di samping berkongsi ciri-ciri histologi dengan neoplasma tiroid konvensional, neoplasma sel Hurthle mungkin berkongsi perubahan genetik yang sama di peringkat awal pembentukan tumor folikular.</span></p>\r\n', 1),
(951, 946, '<p>\r\n	testingtestingtestingtesting</p>\r\n', 1),
(954, 949, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Keupayaan <em>Centella asiatica</em> (CA) untuk bertindak sebagai antioksidan dan agen anti-radang telah banyak dilaporkan. Namun begitu, kaedah pengekstrakan CA untuk memperoleh hasil yang terbaik masih dipersoalkan. Dalam kajian ini, kami menilai tiga kaedah pengekstrakan CA dan membuat perbandingan ekstrak dari segi aktiviti antioksidan dan anti-radang, dan juga kandungan sebatian bioaktif, <em>asiaticosid</em>e dan <em>madecassoside</em>. <em>Centella asiatica</em> diekstrak menggunakan pelarut etanol, metanol dan juga air. Kandungan sebatian fenolik ekstrak diukur menggunakan kaedah reagen Folin-Ciocalteu. Kandungan <em>asiaticoside</em> dan <em>madecassoside </em>ditentukan dengan kaedah HPLC. Aktiviti antioksidan diukur dengan asai <em>2,2-diphenyl-1-picrylhydrazyl</em> (DPPH) dan asai penurunan kuasa <em>Ferric Reducing Antioxidant Power </em>(FRAP). Aktiviti anti-radang ditentukan dengan kebolehan ekstrak untuk merencatkan enzim tapakjalan keradangan, COX-1 dan COX-2, serta kebolehan ekstrak melindungi sel fibroblas aruhan <em>12-O-tetradecanoylphorbol-13-acetate</em> (TPA) daripada menghasilkan prostaglandin E<sub>2</sub> (PGE<sub>2 </sub>). Hasil kajian menunjukkan aras sebatian fenolik, <em>asiaticoside</em> dan <em>madecassoside</em> tertinggi dalam ekstrak etanol, diikuti metanol dan esktrak akues (masing-masing 17.76 g/100g, 15.52 g/100g, 13.16 g/100g untuk sebatian fenolik, 42.86 mg/g, 36.37 mg/g, 2.82 mg/g untuk <em>asiaticoside</em> and 18.66 mg/g, 15.87 mg/g, 3.75 mg/g untuk <em>madecassoside</em>). Ketiga-tiga ekstrak menunjukkan aktiviti antioksidan sederhana berbanding kawalan positif. Kesemua ekstrak, <em>asiaticoside</em> dan <em>madecassoside</em> merencat COX-1 dan COX-2 dan menyekat penghasilan PGE<sub>2&nbsp; </sub>-aruhan TPA. Ekstrak etanol dan metanol merupakan perencat COX yang lebih kuat dan lebih poten daripada ekstrak akues. Oleh itu, walaupun ekstrak akues menunjukkan kebolehan antioksidan yang lebih tinggi, dari segi aktiviti anti-radang,&nbsp; pelarut hidrofobik iaitu etanol dan metanol ternyata lebih baik untuk mengekstrak <em>Centella asiatica</em>.&nbsp;</p>\r\n', 1),
(961, 956, '<p>\r\n	Kajian ini telah dijalankan untuk menguji keberkesanan ceramah demam denggi untuk meningkatkan pengetahuan pengamal perubatan. Ini adalah satu kajian deskriptif keratan rentas yang terdiri daripada penilaian ujian pra dan pasca. Perbandingan data telah dilakukan dengan ujian McNemar dan ujian T bebas. Kita telah mendapat peningkatan yang ketara dalam bahagian pengetahuan untuk semua pengamal perubatan. Skor ujian pra a adalah lebih tinggi di kalangan doktor berbanding dengan paramedik. Walau bagaimanapun, skor ujian pos menunjukkan margin perbezaan mengecil, di mana perbezaan skor dalam kumpulan paramedik adalah lebih besar daripada kumpulan doktor. Terdapat peningkatan statistik semua skor pengetahuan selepas ceramah denggi. Peningkatan ini adalah lebih ketara dalam kumpulan paramedik berbanding dengan kumpulan doktor.</p>\r\n', 1),
(964, 959, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Industri penjagaan kulit Malaysia sedang berkembang maju dengan memperlihatkan peningkatan bilangan krim pelindung cahaya matahari yang dihasilkan setiap tahun. Kekurangan peraturan dan kawalan dalam aspek penjagaan kesihatan kulit menyebabkan ketidaktentuan kualiti krim pelindung cahaya matahari yang dihasilkan. Objektif kajian ini adalah untuk menentukan keberkesanan dan keselamatan krim pelindung cahaya matahari tempatan berbanding dengan krim dari luar negara Malaysia serta kesan penambahan antioksidan asli ke dalam krim-krim tersebut. Tiga krim tempatan dan tiga krim luar negara telah dikaji dengan menggunakan kaedah COLIPA yang dimodifikasi untuk menentukan faktor pencegahan ultraviolet A in vitro (UVAPF) 2009. Sampel krim disapu pada plat polimetilmetakrilat (PMMA) yang kasar di mana penyerapan cahaya dibaca dengan spektrofotometer sebelum dan selepas dua jam pendedahan kepada radiasi cahaya untuk menentukan faktor pencegahan cahaya (SPF), UVAPF dan fotostabiliti. Kaedah yang sama diulangi dengan menambahkan krim pekat vitamin C dan E yang terdapat dalam pasaran. Krim dari luar negara didapati mempunyai label SPF yang jitu (p=0.009) manakala krim tempatan adalah lebih stabil terhadap cahaya (p=0.003). Walau bagaimanapun, kedua-dua krim tidak mempunyai UVAPF yang cukup (p=0.471). Penambahan vitamin C meningkatkan nilai SPF (p=0.04) kedua-dua krim manakala vitamin E meningkatkan fotostabiliti (p=0.000) mereka. Penambahan kombinasi vitamin C dan E kepada krim pelindung cahaya matahari dengan menariknya tidak mengubah nilai SPF dan UVAPF berbanding&nbsp; dengan penambahan satu vitamin secara berasingan. Yang pentingnya, kombinasi vitamin menurunkan fotostabiliti (p=0.002) krim berbanding dengan penambahan vitamin E sahaja.&nbsp; Kesimpulannya, pelindungan terhadap cahaya terbaik dicapai melalui penggunaan krim pelindung cahaya bersama dengan krim yang mengandungi hanya salah satu vitamin C atau E sahaja.</p>\r\n', 1),
(966, 961, '<p>\r\n	Kesesakan di Jabatan Kecemasan telah menjadi masalah yang lama. Penutupan katil hospital atau blok akses ke wad adalah punca tunggal yang paling penting yang menyebabkan kesesakan di Jabatan Kecemasan. Ini boleh di gandakan lagi dalam keadaan peningkatan pesakit secara mendadak contohnya dalam situasi bencana. Respons lonjakan melibatkan reaksi yang besar termasuk melaksanakan pengukuran afirmatif bagi menangani isu ini.</p>\r\n', 1),
(973, 968, '<p>\r\n	Bandar-bandar bandar di dunia mempunyai sinonim yang mempunyai banyak kecederaan dada sama ada dari kemalangan jalan raya atau dari luka ditikam oleh percubaan pembunuhan kejam atau membunuh diri. Apabila pesakit datang ke jabatan kecemasan kami, kami bergegas untuk maksimum usaha untuk menyelamatkan nyawa pesakit. Kami membentangkan laporan kes pesakit yang dikemukakan kepada Jabatan Kecemasan Hospital Raja Permaisuri Bainun, Ipoh, Perak, dengan kecederaan dada tikam yang mempunyai komplikasi embolisasi trombus.</p>\r\n', 1),
(976, 971, '<p>\r\n	&nbsp;</p>\r\n<p style="margin-left:1.0cm;">\r\n	Rawatan haemodialysis melibatkan perubahan pada kecairan darah. Kesan rawatan tersebut ke atas tekanan mata (IOP) dan ketebalan tengah kornea (CCT) masih kurang difahami.&nbsp; Tujuan penyelidikan ini adalah untuk mengkaji kesan haemodialisis terhadap tekanan mata dan ketebalan tengah kornea. Kajian ini telah melibatkan sebanyak 50 mata daripada 50 pesakit yang menjalani rawatan haemodialysis iaitu menjalani pemeriksaan mata, di mana tekanan mata dan ketebalan tengah kornea diukur sebelum dan selepas rawatan haemodialisis. Perubahan berat badan sebelum dan selepas haemodialysis juga diukur. Purata tekanan mata meningkat daripada 14.36 &plusmn; 3.23 mmHg sebelum haemodialyis kepada 14.46 &plusmn; 3.30 mmHg selepas haemodialysis (paired t test, p = 0.798). Purata ketebalan tengah kornea pula berkurang daripada 537.0 &plusmn; 38.9 kepada 532.5 &plusmn; 36.2 &micro;m (paired t test, p = 0.004). Bagaimanapun, tiada korelasi yang signifikan diantara perubahan ketabalan tengah kornea dengan perubahan berat badan. Hasil yang sama dapat dilihat pada korelasi&nbsp; diantara perubahan tekanan mata&nbsp; dengan perubahan ketebalan kornea. Kesimpulan kajian ini iailah rawatan&nbsp; haemodialysis merubah ketebalan tengah kornea, tetapi tidak&nbsp; memberi perubahan kepada tekanan mata.</p>\r\n', 1),
(977, 972, '<p>\r\n	Seni Silat merupakan salah satu daripada beberapa jenis seni pertahanan yang diamalkan oleh Kepulauan Melayu dan Asia Tenggara. Kejuaraan Silat Kebangsaan Ke-16 Kuala Lumpur 2012 telah diadakan dari 24 hingga 29 April 2012 di Stadium Badminton Cheras Kuala Lumpur. Objektif kajian ini adalah untuk menentukan kekerapan, jenis dan corak kecederaan yang berlaku dalam kejuaraan silat tersebut. Kajian ini merupakan kajian &lsquo;retrospektif&rdquo; menggunakan borang PHC yang selaras atau seragam. Semua jenis kecederaan yang dialami oleh peserta sepanjang kejohanan tersebut telah didokumenkan. Jumlah peserta yang terlibat dalam kejohanan ini adalah 158 (114 lelaki dan 44 wanita). Sebanyak 51.6% peserta telah mengalami kecederaan semasa berlangsungnya kejohanan ini. Kecederaan anggota badan yang paling kerap adalah melibatkan anggota atas dan anggota bawah. Jenis-jenis kecederaan yang dialami melibatkan bengkak/hematoma, otot terseliuh, kepatahan,terkehel dan lain-lain. Kebanyakannya adalah kecederaan ringan dan tidak memerlukan rawatan di hospital. Kajian ini juga telah mengenalpasti bahawa kecederaan tisu merupakan antara kecederaan yang paling kerap di kalangan peserta silat. Walau bagaimanapun, kecederaan serius jarang atau tidak berlaku. Pengawasan yang ketat terhadap peraturan kejohanan, latihan yang berterusan dan penggunaan peralatan perlindungan dipercayai menyumbang kepada ketiadaan kecederaan yang teruk atau serius.</p>\r\n', 1),
(979, 974, NULL, NULL),
(980, 975, '<p>\r\n	Madu adalah bahan semulajadi yang dihasilkan oleh lebah madu, <em>Apis mellifera</em>, berpunca dari madu yang diambil dari bunga yang berkembang atau cecair dari pokok dan tumbuhan yang dikenali sebagai <em>honeydews</em>. Ia adalah larutan tepu gula, yang kaya dengan protein, mineral, vitamin, asid organik dan polifenol. Madu mempunyai pelbagai khasiat, sifat penyembuhan dan profilaktik disebabkan oleh komponen-komponen yang terdapat dalam madu. Antara ciri-ciri kesihatan bermanfaat madu termasuk penyembuhan luka, antimikrob, antioksidan dan potensi anti-radang. Kertas kerja ini adalah hasil kajian terkumpul berkaitan komposisi nutrien, antioksidan dan kesan terapeutik madu dengan penekanan kepada madu di Malaysia.</p>\r\n', 1),
(981, 976, '<p>\r\n	Seorang pelajar berusia 17 tahun berketurunan Indonesia mengadu tentang kehilangan penglihatan di kedua- dua belah mata selama 2 minggu. Tiada kontak dengan pesakit tuberculosis (TB) dilaporkan dan ibu- bapanya menyatakan pesakit mendapat imunisasi lengkap termasuk BCG. Walaubagaimanapun, tida parut kesan BCG kelihatan. Penglihatan peakit adalah 6/36 dan 6/60 untuk mata kanan dan kiri. Ruang hadapan dan vitreous mata tiada radangan. Pemeriksaan funduskopi menyerlahkan vaskulitits retina, kebocoran saluran darah, vena tersumbat, saluran darah baru dan macula bengkak. Angiografi menggunakan fluorescein mengesahkan kawasan kapilari yang tidak mendapat perfusi. Ujian Mantoux adalah positif dan pesakit diberikan ubat anti- TB. Ujian HIV adalah negatif. Tiga hari kemudian, ubat antiradang diberikan. Pesakit juga menerima laser di kedua- dua belah&nbsp; matanya. Tiada radangan vitrius sepanjang pesakit menerima rawatan.</p>\r\n', 1),
(982, 977, '<p>\r\n	Kami melaporkan kes seorang lelaki berusia 37 tahun yang merupakan seorang perokok, dating dengan kehilangan medan penglihatan sebelah atas mata kanan secara tiba- tiba tanpa kesakitan. Beliau juga melaporkan episod batuk kering yang tidak respon pada antibiotik dan juga anti- tuberkulosis. Penglihatan beliau hanya setakat mengira jari. Terdapat rekangan retina &lsquo;bullous&rsquo; pada pemeriksaan. Pengimejan radiologi menunjukkan legapan pada peparu, otak dan mata kanan. Bronkoskopi dan biopsy mengesahkan kanser peparu. Kes ini menggambarkan kanser peparu yang &lsquo;bersembunyi&rsquo; di sebalik rekangan retina. Ia juga menekankan kepentingan untuk mensyaki kanser peparu sebagai diagnosa tambahan untuk kes suspisi tuberkulosis di kalangan perokok.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
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(992, 968, 'Kecederaan tusukan dada dianggap sinonim di bandaraya adalah disebabkan kemalangan atau kejadian keganasan. Kecederaan sebegini boleh menyebabkan kematian di tempat kejadian atau komplikasi berpanjangan. Kami melaporkan kes seorang lelaki berumur 39 tahun yang tiba di Jabatan Kecemasan Hospital Raja Permaisuri Bainun, Ipoh, Perak selepas ditikam di dada. Tikaman itu menyebabkan kecederaan pada jantung yang mengakibatkan pembentukkan thrombus. Pesakit juga mengalami hemotoraks di sebelah kiri dan hemoperikardium. Darah daripada hemotoraks ini disalurkan keluar dengan menggunakan tiub dada. Pesakit keluar daripada hospital atas risiko sendiri dan kemudiannya tiba semula di Jabatan Kecemasan mengadu kehilangan penglihatan. Ini disebabkan oleh trombus yang terkeluar dan embolisasi ke arteri retinal menyebabkan amaurosis fugax. Pesakit dirawat dengan aspirin 150 mg dan kemudian symptom beliau beransur pulih.', 1),
(993, 987, '<p>\r\n	Pemulihan fungsi fizikal pesakit selepas strok boleh menentukan prognosis jangka masa panjang mereka. Selain kaedah rehabilitasi, terdapat factor-faktor lain yang boleh mempengaruhi tahap pemulihan ini. Kami menilai tahap pemulihan fizikal jangka pendek pesakit strok iskemik yang mendapat rawatan di hospital tertier serta factor-faktor yang mempengaruhinya. Ini dapat mengesyorkan rawatan strok selepas hospital. METODOLOGI Kajian pemerhatian dijalankan selama 3 bulan. Indeks Bathel yang telah diubah suai (MBI) telah digunakan untuk menilai tahap fungsi fizikal dan kertas soal selidik &ldquo;Patient Health-9&rdquo; (PHQ-9) untuk mengesan tahap kemurungan pesakit pasca strok. KEPUTUSAN Sejumlah 46 pesakit strok telah dimasukkan ke dalam kajian ini. Walaubagaimanapun, hanya 37 pesakit berjaya mendapat pemeriksaan susulan dan dianalisa. Purata umur bagi pesakit adalah 67.2 (&plusmn;11) tahun. Melayu merupakan kaum majority (50%) diikuti oleh kaum Cina (41.3%) dan India (8.7%). Tekanan darah tinggi adalah faktor risiko paling tinggi (89.1%) diikuti oleh masalah kolesterol (65.2%) dan diabetis (63.0%). Purata kadar perbezaan MBI ialah 45.2 (SD 27.0) dengan median MBI 17.0 (IQR 33.0) pada permulaan kajian dibandingkan dengan 85.0 (IQR 42.0) selepas 3 bulan (p&lt;0.001,CI 35.98,55.45). Prevalen kemurungan selepas strok (PHQ-9&ge;10) adalah sebanyak 21.6%. Kami mendapati bahawa pesakit yang mempunyai kemurungan pada masa 3 bulan dan mereka yang pernah mengalami strok sebelum ini mempunyai pemulihan fungsi yang lebih rendah (p &lt; 0.035). KESIMPULAN Pemulihan yang nyata dari segi fungsi fizikal telah ditunjukkan oleh pesakit strok dalam 3 bulan pertama selepas strok. Faktor seperti kemurungan dan pernah mengalami strok boleh mempengaruhi kadar pemulihan, oleh itu perlu untuk di saring pada peringkat awal.</p>\r\n', 1),
(997, 991, '<div>\r\n	Keupayaan Centella asiatica (CA) untuk bertindak sebagai antioksidan dan agen anti-radang telah banyak dilaporkan. Namun begitu, kaedah pengekstrakan CA untuk memperoleh hasil yang terbaik masih dipersoalkan. Dalam kajian ini, kami menilai tiga kaedah pengekstrakan CA dan membuat perbandingan ekstrak dari segi aktiviti antioksidan dan anti-radang, dan juga kandungan sebatian bioaktif, asiaticoside dan madecassoside. Centella asiatica diekstrak menggunakan pelarut etanol, metanol dan juga air. Kandungan sebatian fenolik ekstrak diukur menggunakan kaedah reagen Folin-Ciocalteu. Kandungan asiaticoside dan madecassoside ditentukan dengan kaedah HPLC. Aktiviti antioksidan diukur dengan asai 2,2-diphenyl-1-picrylhydrazyl (DPPH) dan asai penurunan kuasa Ferric Reducing Antioxidant Power (FRAP). Aktiviti anti-radang ditentukan dengan kebolehan ekstrak untuk merencatkan enzim tapakjalan keradangan, COX-1 dan COX-2, serta kebolehan ekstrak melindungi sel fibroblas aruhan 12-O-tetradecanoylphorbol-13-acetate (TPA) daripada menghasilkan prostaglandin E2 (PGE2 ). Hasil kajian menunjukkan aras sebatian fenolik, asiaticoside dan madecassoside tertinggi dalam ekstrak etanol, diikuti metanol dan esktrak akues (masing-masing 17.76 g/100g, 15.52 g/100g, 13.16 g/100g untuk sebatian fenolik, 42.86 mg/g, 36.37 mg/g, 2.82 mg/g untuk asiaticoside and 18.66 mg/g, 15.87 mg/g, 3.75 mg/g untuk madecassoside). Ketiga-tiga ekstrak menunjukkan aktiviti antioksidan sederhana berbanding kawalan positif. Kesemua ekstrak, asiaticoside dan madecassoside merencat COX-1 dan COX-2 dan menyekat penghasilan PGE2 -aruhan TPA. Ekstrak etanol dan metanol merupakan perencat COX yang lebih kuat dan lebih poten daripada ekstrak akues. Oleh itu, walaupun ekstrak akues menunjukkan kebolehan antioksidan yang lebih tinggi, dari segi aktiviti anti-radang, pelarut hidrofobik iaitu etanol dan metanol ternyata lebih baik untuk mengekstrak Centella asiatica.</div>\r\n', 1),
(1000, 994, '<div>\r\n	Adalah lazim untuk seseorang ibubapa mengalami stres apabila menjalankan peranan sebagai seorang ibu/bapa (Hoekstra-Weebers et al. 1998). Terdapat pelbagai punca stres dan stres in bergantung juga kepada fasa rawatan dan peringkat kemoterapi (Sawyer et al. 2000). Jika stres ini tidak ditangani dengan elok, maka ianya dapat mendatang kesan terhadap aspek emosi dan sosial seseorang kanak-kanak dalam menerima diagnosa kanser selain daripada penerimaan rawatan yang diberi (Sawyer et al. 1993). Kajian ini bertujuan untuk mengenalpasti tahap stres dikalangan ibubapa, faktor-faktor risiko yang berkaitan dengan tahap stres yang tinggi, disamping jenis mekanisme yang digunakan oleh ibubapa untuk menangani stres tersebut. Seramai 117 ibu/bapa terlibat didalam kajian keratan lintang ini yang telah dijalankan di Unit Pediatrik Hematologi/Onkologi di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) dalam jangkamasa dua tahun. Ibu/bapa kepada pesakit yang berusia 12 tahun ke bawah telah diminta menjawab dua jenis borang soal selidik, iaitu Parenting Stress Index/Short Form (PSI/SF) dan Coping Inventory for Stressful Situation (CISS). Keputusan menunjukkan bahawa purata jumlah skor PSI untuk tahap stres dikalangan ibu/bapa ini ialah 91.5&plusmn;21.1(95% CI). Sejumlah 27.3% daripada golongan ibu/bapa mencatat tahap jumlah skor PSI yang tinggi (definisi ialah jumlah skor PSI &ge;103, iaitu &ge;&ge;julat ke-75). Majoriti daripada ibu/bapa menggunakan mekanisme &#39;task-oriented&#39; untuk menangani stres yang dialami. Berdasarkan analisa multiple logistic regression, mekanisme emotion-oriented merupakan satu-satunya faktor risiko yang signifikan bagi tahap jumlah skor PSI yang tinggi. Seseorang ibu/bapa yang menggunakan mekanisme &#39;emotion-oriented&#39; adalah 7.1 kali ganda lebih (julat konfidens 95%: 1.2 hingga 41.4) berkemungkinan untuk mencatat jumlah skor PSI yang tinggi jika dibandingkan dengan mereka yang menggunakan mekanisme yang lain. Dengan mengenalpasti golongan ibubapa yang berisiko tinggi ini, kaunseling dan sokongan psikologi akan dapat diberikan lebih awal supaya tahap stres mereka akan dapat ditangani dengan lebih efektif.</div>\r\n', 1),
(1006, 1000, '<p>\r\n	Industri penjagaan kulit Malaysia sedang berkembang maju dengan memperlihatkan peningkatan bilangan krim pelindung cahaya matahari yang dihasilkan setiap tahun. Kekurangan peraturan dan kawalan dalam aspek penjagaan kesihatan kulit menyebabkan ketidaktentuan kualiti krim pelindung cahaya matahari. Objektif kajian ini adalah untuk menentukan keberkesanan dan keselamatan krim pelindung cahaya matahari tempatan berbanding dengan krim antarabangsa serta kesan penambahan antioksidan asli ke dalam krim-krim tersebut. Tiga krim tempatan dan tiga krim luar negara telah dikaji untuk menentukan faktor pencegahan ultraviolet A (UVAPF), faktor pencegahan cahaya (SPF) dan fotostabiliti secara in vitro. Sampel krim disapu pada plat polimetilmetakrilat yang kasar di mana penyerapan cahaya dibaca dengan spektrofotometer sebelum dan selepas dua jam pendedahan kepada radiasi cahaya. Kaedah yang sama diulangi dengan menambahkan krim pekat vitamin C dan E yang terdapat dalam pasaran. Krim dari luar negara didapati mempunyai label SPF yang jitu (p=0.009) manakala krim tempatan adalah lebih stabil terhadap cahaya (p=0.003). Walau bagaimanapun, kedua-dua krim tidak mempunyai UVAPF yang cukup (p=0.471). Penambahan vitamin C meningkatkan nilai SPF (p=0.04) kedua-dua krim manakala vitamin E meningkatkan fotostabiliti (p=0.000) mereka. Penambahan kombinasi vitamin C dan E kepada krim pelindung cahaya matahari dengan menariknya tidak mengubah nilai SPF dan UVAPF berbanding dengan penambahan satu vitamin secara berasingan. Yang pentingnya, kombinasi vitamin menurunkan fotostabiliti (p=0.002) krim berbanding dengan penambahan vitamin E sahaja. Kesimpulannya, pelindungan terhadap cahaya terbaik dicapai melalui penggunaan krim pelindung cahaya bersama dengan krim yang mengandungi hanya salah satu vitamin C atau E sahaja.</p>\r\n', 1),
(1011, 1005, '<p>\r\n	Seorang pelajar berusia 17 tahun berketurunan Indonesia mengadu kehilangan penglihatan di kedua-dua belah mata selama dua minggu. Tiada kontak dengan pesakit tuberculosis (TB) dilaporkan dan ibu-bapanya menyatakan pesakit mendapat imunisasi lengkap termasuk BCG. Walaubagaimanapun, tiada kesan parut BCG kelihatan. Penglihatan pesakit adalah 6/36 dan 6/60 untuk mata kanan dan kiri. Ruang hadapan dan vitreous mata tiada radangan. Pemeriksaan funduskopi menyerlahkan vaskulitits retina, kebocoran saluran darah, vena tersumbat, saluran darah baru dan makula bengkak. Angiografi menggunakan fluorescein mengesahkan kawasan kapilari yang tidak mendapat perfusi. Ujian Mantoux adalah positif dan pesakit diberikan ubat anti- TB. Ujian HIV adalah negatif. Tiga hari kemudian, ubat antiradang diberikan. Pesakit juga menerima laser di kedua-dua belah matanya. Tiada terdapat radangan vitrius sepanjang pesakit menerima rawatan.</p>\r\n', 1),
(1014, 1008, '<p>\r\n	Ascariasis adalah salah satu jangkitan cacing di Malaysia, lebih-lebih lagi di kawasan pendalaman. Kebiasaannnya ascariasis lumbricoides yang masuk dan tinggal di dalam salur pencernaan manusia tidak akan menimbulkan apa-apa simptom. Sekiranya cacing itu masuk ke dalam saluran hempedu, ia akan menyebabkan komplikasi yang dinamakan askariasis biliari. Satu kes askariasis biliari yang menunjukan simptom-simptom seperti kolik biliari akan dibincangkan. Pesakit ini telah datang ke Hospital Daerah mengadu sakit pada bahagian atas dan kanan perut selama dua hari. Siasatan awal pemeriksaan dan ujian makmal menunjukkan pesakit ini mempunyai simptom-simptom seperti acute cholecystitis dan pesakit telah ditahan wad dan dirawat dengan antibiotik. Disebabkan simptom yang bertambah buruk, pesakit ini dirujuk ke hospital besar yang berdekatan.Pesakit ini telah menderita akibat infeksi cacing dibahagian hempedu setelah di dikonfirmasikan oleh ultrasound HBS, CT-Scan abdomen dan ERCP, dimana cacing tersebut dikeluarkan melalui prosedur endoskopi. Setelah menjalani pemeriksaan dan prosedur, pesakit telah sembuh dan dibenarkan keluar dari wad.</p>\r\n', 1),
(1020, 1014, '<p>\r\n	Levonorgestrel (LNG) sememangnya dikenali sebagai pil pencegahan kehamilan kecemasan (EC) yang selamat and berkesan. Namun begitu, kes kehamilan luar rahim akibat daripada kegagalan LNG EC pernah dilaporkan. Insiden kehamilan luar rahim yang tepat sukar ditentukan akibat kemudahan untuk mendapatkan LNG EC daripada farmasi tanpa preskripsi. Kami melaporkan satu kes kehamilan luar rahim seorang pesakit perempuan tidak berkahwin yang berumur 18 tahun. Beliau telah mengambil LNG EC dalam jangkamasa 48 jam selepas persetubuhan. Beliau dihantar ke jabatan kecemasan pada minggu kelapan tempoh hamil dengan abdomen akut dan renjatan hipovolemik. Pembedahan laparotomi mengesahkan kehamilan di luar rahim yang sudah pecah di bahagian tiub fallopio dan salpingektomi telah dilaksanakan. Pesakit tersebut dibenarkan pulang selepas dua hari. Kami ingin menekankan kesan sampingan dan membincangkan perkaitan kehamilan luar rahim selepas penggunaan LNG EC.</p>\r\n', 1),
(1026, 1020, '<p>\r\n	Megakaryoblastik leukemia adalah sejenis myeloid leukemia akut yang sering dihidapi bayi penghidap sindrom Down. Megakaryoblastik leukemia dengan translokasi t(1;22)(p13;q13) adalah sejenis myeloid leukemia akut yang jarang ditemui dan meliputi hanya &lt;1% bagi semua kes. Ia dilaporkan berlaku hanya kepada bayi penghidap megakaryoblastik leukemia yang tidak mengalami sindrom Down. Ia lebih melibatkan bayi perempuan dan translokasi sitogenetik ini dilaporkan mempunyai impak yang buruk kepada prognosis pesakit. Kami melaporkan kes yang jarang ditemui ini berlaku kepada seorang bayi perempuan berumur 9 bulan yang menunjukkan simptom tanda-tanda lebam, pembengkakan hati dan limpa yang serius dan pembengkakan kelenjar limfa servikal dan inguinal. Darah periferi menunjukkan anemia yang serius, ovalositosis, trombositopenia dan sel darah putih yang tinggi. Sum-sum tulang menunjukkan kepadatan sel-sel blast yang mempunyai ratio nukleus-sitoplasmic yang tinggi dan sitoplasm tanpa granul dan blebs. Ujian peroksidase adalah negatif. Ujian imunofenotip menunjukkan ekspresi penanda CD117, CD 13, CD33 dan CD61 yang mengesahkan diagnosa megakaryoblastik leukemia akut. Menariknya, ujian sitogenetik menunjukkan kehadiran translokasi t(1;22) yang dilaporkan berlaku kepada bayi yang tidak mengalami sindrom Down berlaku pada kes ini. Pesakit ini menerima rawatan rejim kemoterapi mengikut protokol AML trial 15 ADE dan sebagai kesannya telah mengalami neutropenik sepsis yang serius dan masalah pernafasan yang memerlukan bantuan pernafasan dan faktor stimulasi koloni granulosit (G-CSF). Walaubagaimanapun, pesakit tidak menghadiri sesi temujanji seterusnya setelah sembuh dan beberapa bulan kemudian kembali dengan penyakit myeloid lukemia akut berulang.</p>\r\n', 1),
(1027, 961, 'Kesesakan Jabatan Kecemasan (EDOC) telah menjadi masalah yang tidak pernah selesai. Ia ditakrifkan sebagai permintaan perkhidmatan kecemasan melebihi keupayaan Jabatan Kecemasan (ED) untuk menyediakan penjagaan yang berkualiti dalam kadar masa yang sesuai.1,2 Penutupan wad Hospital atau penyekatan akses untuk menahan kemasukan pesakit adalah salah satu punca yang penting dalam menyebabkan kesesakan  Jabatan Kecemasan.3 Keadaan ini menjadi semakin teruk dalam situasi bencana. Kaedah yang tertentu perlu dirangka untuk mengatasi masalah ini. Langkah-langkah dalam menguruskan EDOC adalah: 1.Mengenalpasti EDOC, 2.Memulakan tindakan, 3. Membaikpulih aliran pesakit, 4. Menetapkan matlamat rawatan pesakit dan 5. Menggerakkan sebuah pasukan untuk Pendahuluan triage atau laluan cepat.\r\n	', 1),
(1029, 1022, '<p>\r\n	Obesiti telah menjadi satu isu kesihatan yang utama di Malaysia. Obesiti berupaya menganggu keihatan fizikal dan mental. Dari segi mental, beberapa kajian mendapati kemurungan adalah lazim dikalangan mereka yang obese. Walaubagaimanapun, tahap kemurungan adalah berbeza mengikut&nbsp; individu. Objektif kajian ini adalah untuk menilai hubungan di antara obesity dan kemurungan.&nbsp; Satu kajian quantitative secara keratan rentas telah dijalankan dengan mengunakan persampelan mudah atau &lsquo;<em>convenient</em> <em>sampeling&rsquo;. </em>Responden yang memenuhi criteria saperti warganegara Malaysia yang berkerja di Malaysia dengan indeks jisim badan 30 dan keatas telah dipilih secara rawak. Soal selidik berstruktur yang terbahagi kepada lima bahagian telah digunakan untuk mengumpul data. Sejumlah 60 puluh orang telah mengambil bahagian dalam kajian ini. Data dianalisis menggunakan perisian SPSS versi 20. Keputusan kajian menunjukkan bahawa kadar kemurungan dikalangan responden adalah 23.3%. Tiada perbezaan signifikan dari segi kemurungan antara lelaki dan wanita yang mengalami masaalah obesiti</p>\r\n<p>\r\n	(x<sup>2 </sup>= 0.3568, p=0.059). Terdapat kaitan antara kemurungan dan langkah aktif yang diambil untuk mengurangkan berat badan (x<sup>2</sup>=5.963, p=0.05). Kajian ini mendapati tidak semua individu yang obese akan mengalami kemurungan. Walaubagaimanapun kesedaran orang ramai tentang bahaya obesiti adalah penting kerana pengesanan awal dapat menghindari kesan negative yang tidak diingini dikalangan penduduk yang berkerja di Malaysia</p>\r\n', 1),
(1036, 1029, 'Kajian ini bertujuan untuk menentukan faktor-faktor yang berkait dengan penyakit ginjal kronik tahap 3b di kalangan pesakit diabetes jenis 2 yang dirawat di pusat perubatan primer, khususnya peranan dos ubatan yang menyekat angiotensin.  Kajian ini merupakan kajian kes-kawalan pilot yang tidak dipadankan dan dijalankan di sebuah pusat pengajaran perubatan primer.  Data klinikal 25 kes pesakit dengan penyakit ginjal kronik tahap 3b (GFR 30-45ml/min/1.73m2) dalam tahun 2012 dipilih sebagai kes untuk kajian ini.  Di samping itu, 103 pesakit diabetes yang mempunyai GFR lebih daripada 45ml/min/1.73m2 dalam tahun 2012 dipilih sebagai kawalan.  Sampel dipilih secara rawak sistematik.  Data pesakit diperolehi melalui rekod diabetes pesakit, sistem maklumat kesihatan komputer, dan fail perubatan pesakit.  Analisis univariat menggunakan ujian chi square, t-test, Fisher’s exact test dan Mann-Whitney U-test.  Regresi logistik digunakan untuk menentukan faktor-faktor yang berkait dengan pembentukan penyakit ginjal kronik tahap 3b.  Kes dan kawalan berbeza dari segi umur, jangkamasa diabetes, penggunaan dan dos ubat penyekat angiotensin, tekanan darah sistolik dan GFR permulaan.  Regresi logistik menunjukkan bahawa tekanan darah sistolik (Adjusted OR= 1.08, 95% CI= 1.02-1.14, p=0.013) and GFR permulaan (Adjusted OR= 0.90, 95% CI= 0.85-0.95, p<0.001) berkait secara signifikan dengan pembentukan penyakit ginjal kronik tahap 3b di kalangan pesakit diabetes.  Penggunaan dos ubat penyekat angiotensin yang maksimum memperlihatkan kesan protektif.  (Adjusted OR= 0.14, 95% CI=0.85-0.95, p=0.025)  Kajian ini menyokong keperluan kawalan tekanan darah sistolik yang baik untuk melambatkan kemerosotan fungsi ginjal.  Dos ubat penyekat angiotensin perlu dioptimumkan di kalangan pesakit-pesakit ini.  ', 1),
(1040, 1033, NULL, NULL),
(1042, 1035, '<p>\r\n	Madu adalah bahan semulajadi yang dihasilkan oleh lebah madu, Apis mellifera, berpunca daripada madu yang diambil dari bunga yang berkembang atau cecair dari pokok dan tumbuhan yang dikenali sebagai madu nektar dan madu serangga masing-masing. Ia adalah larutan tepu gula, yang kaya dengan protein, mineral, vitamin, asid organik dan polifenol. Madu mempunyai pelbagai khasiat, sifat penyembuhan dan profilaktik disebabkan oleh komponen-komponen yang terkandung di dalamnya. Madu mempunyai beberapa khasiat kesihatan seperti penyembuhan luka, antimikrob, antioksidan dan potensi anti-radang. Ulasan ini adalah berkaitan komposisi nutrien, antioksidan dan kesan terapeutik madu dengan penekanan kepada madu di Malaysia.</p>\r\n', 1),
(1044, 1037, '<p>\r\n	Pemulihan fungsi fizikal pesakit selepas strok boleh menentukan prognosis jangka masa panjang mereka. Selain kaedah rehabilitasi, terdapat faktor-faktor lain yang boleh mempengaruhi tahap pemulihan ini. Kami menilai tahap pemulihan fizikal jangka pendek pesakit strok iskemik yang mendapat rawatan di hospital tertier serta faktor-faktor yang mempengaruhinya. Ini dapat mengesyorkan rawatan lanjut strok selepas kemasukan hospital. Kajian pemerhatian dijalankan selama tiga bulan. Indeks Bathel yang telah diubah suai (MBI) digunakan untuk menilai tahap fungsi fizikal dan kertas soal selidik &ldquo;Patient Health Questionnaire-9&rdquo; (PHQ-9) untuk mengesan tahap kemurungan pesakit pasca strok. Sejumlah 46 pesakit strok terlibat di dalam kajian ini. Walaubagaimanapun, hanya 37 pesakit berjaya mendapat pemeriksaan susulan dan dianalisa. Purata umur bagi pesakit adalah 67.2 (&plusmn;11) tahun. Melayu merupakan kaum majority (50%) diikuti dengan kaum Cina (41.3%) dan India (8.7%). Tekanan darah tinggi adalah faktor risiko paling tinggi (89.1%) diikuti dengan masalah kolesterol (65.2%) dan diabetis (63.0%). Purata kadar perbezaan MBI ialah 45.2 (SD 27.0) dengan median MBI 17.0 (IQR 33.0) pada permulaan kajian dibandingkan dengan 85.0 (IQR 42.0) selepas tiga bulan (p&lt;0.001,CI 35.98,55.45). Prevalen kemurungan selepas strok (PHQ-9&ge;10) adalah sebanyak 21.6%. Kami mendapati bahawa pesakit yang mempunyai kemurungan pada masa tiga bulan dan mereka yang pernah mengalami strok sebelum ini mempunyai pemulihan fungsi yang lebih rendah (p&lt;0.035). Pemulihan yang nyata dari segi fungsi fizikal telah ditunjukkan oleh pesakit strok dalam tempoh tiga bulan pertama selepas strok. Faktor seperti kemurungan dan pernah mengalami strok boleh mempengaruhi kadar pemulihan, oleh itu perlu untuk di saring pada peringkat awal.</p>\r\n', 1),
(1048, 1041, '<p>\r\n	Kecederaan tusukan dada dianggap sinonim di bandaraya adalah disebabkan kemalangan atau kejadian keganasan. Kecederaan sebegini boleh menyebabkan kematian di tempat kejadian atau komplikasi berpanjangan. Kami melaporkan kes seorang lelaki berumur 39 tahun yang tiba di Jabatan Kecemasan Hospital Raja Permaisuri Bainun, Ipoh, Perak selepas ditikam di dada. Tikaman itu menyebabkan kecederaan pada jantung yang mengakibatkan pembentukkan thrombus. Pesakit juga mengalami hemotoraks di sebelah kiri dan hemoperikardium. Darah daripada hemotoraks ini disalurkan keluar dengan menggunakan tiub dada. Pesakit keluar daripada hospital atas risiko sendiri dan kemudiannya tiba semula di Jabatan Kecemasan mengadu kehilangan penglihatan. Ini disebabkan oleh trombus yang terkeluar dan embolisasi ke arteri retinal menyebabkan amaurosis fugax. Pesakit dirawat dengan aspirin 150 mg dan kemudian symptom beliau beransur pulih.</p>\r\n', 1),
(1053, 1046, '<p>\r\n	Rasa sakit merupakan suatu perasaan yang sangat kurang enak dan tidak menyenangkan. Wanita hamil akan merasakan kesakitan yang amat sangat semasa melahirkan anak. Walaupun rasa sakit yang amat kuat tidak membawa kemudaratan yang mengancam nyawa, namun ianya boleh meninggalkan kesan neuropsikologi yang ketara. Misalnya kemurungan lepas bersalin adalah lebih kerap dilaporkan bagi mereka yang tidak menerima ubat analgesia dan kesakitan kuat yang dialami semasa bersalin adalah berkait rapat dengan kejadian gangguan-tekanan-pasca-trauma. Pemberian bius epidural semasa bersalin melibatkan pemberian ubat bius setempat di kawasan tulang belakang. Ianya dianggap sebagai rawatan piawai bagi melegakan kesakitan semasa proses bersalin. Kami ingin berkongsi satu kajian kes yang melibatkan seorang pesakit berusia 37 tahun gravida 1 para 0 yang dirawat dengan bius epidural bagi mengawal kesakitan semasa melahirkan anak.</p>\r\n', 1),
(1056, 1049, '<p>\r\n	Produk semulajadi sering dirasakan selamat digunakan kerana ia dianggap suci dan bebas dari sebarang bahan kimia. Walaubagaimanapun, ramai yang tidak menyedari bahawa sebahagian daripada kandungan bahan semulajadi boleh mengakibatkan kesan negatif kepada kulit. Kulit kayu manis dan limau adalah di antara ramuan semulajadi yang kerap digunakan sebagai rawatan tradisional untuk mengubati jerawat. Kedua-dua bahan ini boleh menyebabkan keradangan apabila bersentuhan dengan kulit manusia. Tujuan utama artikel ini ialah untuk menekankan bahawa penggunaan bahan semulajadi tidak semestinya bebas dari sebarang kesan sampingan dan ia perlu diambil kira sebagai penyebab masalah kulit seseorang pesakit yang menggunakannya. Pengamal-pengamal perubatan perlu peka tentang kewujudan masalah ini dan harus menyoal pesakit tentang penggunaan rawatan-rawatan buatan sendiri, kerana pesakit kadang-kala tidak memberikan maklumat ini secara sukarela. Pengenalpastian bahan penyebab dan kaunseling amat penting agar pesakit tidak mengulangi penggunaan bahan tersebut bagi mengelakkan sebarang komplikasi di masa hadapan.</p>\r\n', 1),
(1058, 1051, '<p>\r\n	Kesesakan Jabatan Kecemasan (EDOC) telah menjadi masalah yang tidak pernah selesai. Ia ditakrifkan sebagai permintaan perkhidmatan kecemasan melebihi keupayaan Jabatan Kecemasan (ED) untuk menyediakan penjagaan yang berkualiti dalam kadar masa yang sesuai. Penutupan wad hospital atau penyekatan akses untuk menahan kemasukan pesakit adalah salah satu punca yang penting dalam menyebabkan kesesakan Jabatan Kecemasan. Keadaan ini menjadi semakin teruk dalam situasi bencana. Kaedah yang tertentu perlu dirangka untuk mengatasi masalah ini. Langkah-langkah dalam menguruskan EDOC adalah: 1. Mengenalpasti EDOC, 2. Memulakan tindakan, 3. Membaikpulih aliran pesakit, 4. Menetapkan matlamat rawatan pesakit dan 5. Menggerakkan sebuah pasukan untuk Pendahuluan triage atau laluan cepat.</p>\r\n', 1),
(1060, 1053, '<p>\r\n	Kami melaporkan kes seorang lelaki berusia 37 tahun yang merupakan seorang perokok, datang dengan kehilangan medan penglihatan sebelah atas mata kanan secara tiba-tiba tanpa kesakitan. Beliau juga melaporkan episod batuk kering yang tidak respon pada antibiotik dan juga anti-tuberkulosis. Penglihatan beliau hanya setakat mengira jari. Terdapat rekangan retina &lsquo;bullous&rsquo; pada pemeriksaan. Pengimejan radiologi menunjukkan legapan pada peparu, otak dan mata kanan. Bronkoskopi dan biopsy mengesahkan kanser peparu. Kes ini menggambarkan kanser peparu yang &lsquo;bersembunyi&rsquo; di sebalik rekangan retina. Ia juga menekankan kepentingan untuk melihat kanser peparu sebagai diagnosa tambahan bagi kes disyaki tuberkulosis di kalangan perokok.</p>\r\n', 1),
(1074, 1067, '<p>\r\n	ABSTRAK Talasemia adalah penyakit gangguan genetik heterogenus diakibatkan oleh perubahan sintesis pada satu atau lebih rantaian globin. Jenis yang paling kerap ditemui di Malaysia adalah talasemia-&alpha;, talasemia-&beta;, hemoglobin E dan hemoglobin Constant Spring. Pengesanan awal amat penting bagi mengelakkan kelahiran penyakit hemoglobin yang serius. Seramai 738 penderma darah yang sihat dari Hospital Tengku Ampuan Rahimah, Klang telah disaring menggunakan kaedah cation-exchange high performance liquid chromatography (HPLC) dari Julai hingga September 2010. Semua kes varian hemoglobin seterusnya diproses menggunakan gel electrophoresis pada pH alkali. Hasil kajian menunjukkan penderma darah terdiri daripada bangsa Melayu 370 (56.2%), India 139 (21.1%), Cina 134 (20.4%) and lain-lain 13 (2%). Terdapat 16 (2.3%) trait hemoglobin E, enam (0.9%) hemoglobin E/talasemia-&alpha; dan lima (0.7%) trait talasemia-&beta;. Hemoglobin Constant Spring dan hemoglobin A2 prime dilihat dalam dua (0.3%) kes; dan Hemoglobin Lepore serta varian rantaian alpha dalam satu (0.2%) kes. Walaubagaimanapun talasemia-&alpha; dan talasemia-&beta; hemoglobin A2 normal tidak boleh diabaikan dalam 109 (16.6%) kes dan memerlukan analisa DNA untuk pengenalpastian jenis talasemia tersebut. Saringan talasemia ke atas penderma darah adalah lebih mudah dan berkesan. Oleh yang demikian saringan populasi yang meluas termasuk penderma darah boleh memberi manfaat terhadap program saringan talasemia yang sedia ada di Malaysia.</p>\r\n', 1),
(1077, 1070, '<p>\r\n	Tujuan penyelidikan ini dijalankan ialah untuk menilai tahap pengetahuan masyarakat terhadap faktor risiko, tanda-tanda, rawatan dan sumber maklumat berhubung strok dan Transient Ischaemic Attack, dan seterusnya menjana profil risiko strok and melihat hubungkaitnya dengan pengetahuan mereka. Menggunakan borang kajiselidik yang telah divalidasi dan dicuba, 112 orang telah berjaya disoalselidik pada dua acara kemasyarakatan yang berbeza- Hari Kesedaran Strok dan Hari Terbuka Kemasyarakatan. Orang ramai telah mengisi borang kajiselidik dan kemudian menjalani ujian saringan kesihatan. 100 orang telah layak untuk kajian ini. Maklumat yang dikumpul telah dianalisa menggunakan SPSS versi 20. Keputusan: Secara keseluruhannya, pengetahuan awam didapati sangat lemah. Hanya 35% daripada peserta yang telah dikategorikan sebagai mempunyai pengetahuan yang memuaskan berkaitan tanda-tanda strok (p=0.94) dan 29% mempunyai pengetahuan memuaskan berkaitan faktor risiko (p=0.46). Setelah dibahagikan antara kumpulan risiko, didapati peratusan pengetahuan mengenai strok yang memuaskan adalah: pesakit berisiko tinggi (41%), berisiko sederhana (30%) dan berisiko rendah (26%). Tiada seorangpun peserta yang tahu berkenaan wujudnya rawatan bagi serangan strok akut atau kepentingan rehabilitasi sebagai sebahagian daripada pengurusan strok. Kesimpulan: Kesedaran awam berhubung faktor risiko, tanda-tanda dan pengurusan strok adalah pada tahap yang membimbangkan.</p>\r\n', 1),
(1084, 1077, 'Osteoporosis dikenali sebagai penyakit senyap kerana tidak mempunyai tanda-tanda awal. Ini disebabkan oleh kepadatan tulang yang berkurangan secara perlahan-lahan seiring dengan peningkatan usia. Insiden penyakit ini semakin meningkat setiap tahun di seluruh dunia. Mengukur ketumpatan mineral tulang (BMD) menggunakan densitometry tulang konvensional (DXA) adalah praktikal dalam diagnosis osteoporosis tetapi kosnya adalah tinggi dan tidak dapat dilaksanakan dalam masyarakat. Untuk mengukur ketumpatan tulang “quantitative ultrasound (QUS)” adalah teknik yang agak moden untuk diagnosis osteoporosis. Ianya agak  mudah, konsisten, lebih murah dan kaedah yang selamat berbanding dengan teknik densitometry yang lain. Kedua-dua parameter “quantitative ultrasound (QUS)” kini diukur adalah ultrasound jalur pengecilan (BUA) dan kelajuan bunyi (SOS). QUS juga dapat menjangka risiko patah. Ianya kini digunakan untuk memantau tindak balas kepada rawatan anti-osteoporosis. Kajian in-vitro menunjukkan bahawa indeks QUS berhubungkait dengan BMD, bentuk tulang mikro dan parameter mekanikal. Dengan yang demikian QUS adalah teknik baru untuk penilaian tulang. ', 1),
(1087, 1080, 'Objektif: Tujuan kajian ini adalah untuk menentukan dan menganalisis faktor-faktor yang mempengaruhi paramedik Jabatan Kecemasan Hospital Universiti Kebangsaan Malaysia dalam menggunakan AED . Kajian ini juga menilai pengetahuan, tahap keyakinan dan mengenal pasti kekurangan dalam pengunaan AED.\r\nRekabentuk: Ini adalah satu kajian prospektif rentas ‘cross sectional’ yang dijalankan antara bulan Disember 2013 dan Januari 2014. Paramedik dari Jabatan Kecemasan HUKM telah mendaftar dan dinilai menggunakan soal selidik yang terdiri daripada beberapa bahagian termasuk: . Penilaian pengetahuan , latihan dan amalan.\r\nKeputusan : 53 paramedik telah mengambil bahagian dalam kajian ini. Hanya 62.3 % peserta telah menggunakan AEDs di luar hospital. Hanya 83% peserta mengakui bahawa mereka akan menggunakannya jika diperlukan. Satu korelasi positif yang ketara dilihat antara umur dan pengalaman kerja dengan pengetahuan mengenai AED (p = 0.001 dan p = 0.005 masing-masing) . Graduan institut kerajaan mempunyai pengetahuan yang lebih baik dan tahap keyakinan lebih tinggi daripada graduan institusi swasta ''(p <0.001 ). Korelasi positif statistik yang signifikan juga dilihat antara pengalaman kerja dan tahap keyakinan dalam membuat keputusan untuk menggunakan AED (p = 0.006 ), memohon AED (p = 0.019 ) dan penyelesaian masalah komplikasi yang timbul daripada penggunaan AED (p = 0.002 ). Faktor utama mengurangkan tahap keyakinan AED penggunaan adalah latihan yang terbatas ( 73.6 %) yang menyebabkan keyakinan dikurangkan untuk memulakan penggunaan (45.3 %). 88.6 % bersetuju bahawa latihan adalah penting sebelum sebarang penggunaan AED . 41.5 % menyatakan bahawa AEDs tidak bersedia untuk orang ramai di Malaysia .\r\nKesimpulan: penggunaan AED dan pengetahuan di kalangan paramedik masih rendah. Latihan yang mencukupi adalah penting bagi paramedik.\r\n\r\n', 1),
(1088, 1067, '         Program saringan talasemia telah diwujudkan di Malaysia semenjak tahun 2004. Program ini bertujuan untuk mengurangkan beban akibat penyakit ini dengan mengenal-pasti pembawa talasemia. Walaubagaimanapun, sambutan terhadap aktiviti saringan ini didapati kurang memuaskan kerana kurangnya kesedaran orang ramai akan kepentingan saringan talasemia. Kaedah alternatif adalah menyaring penderma darah. Matlamat kajian ini adalah untuk mengetahui prevalens pembawa talasemia di kalangan penderma darah yang sihat.\r\n	Tujuh ratus tiga puluh lapan penderma darah yang sihat telah disaring di Hospital Tengku Ampuan Rahimah, Klang menggunakan kaedah cation-exchange high performance liquid chromatography (HPLC) dari Julai hingga September 2010. Semua kes varian hemoglobin seterusnya diproses menggunakan gel electrophoresis pada pH alkali.\r\n         Hasil kajian menunjukkan penderma darah terdiri daripada bangsa Melayu 413 (56%), India 162 (22%), Cina 148 (20%) and lain-lain 15 (2%). Terdapat 19 (2.6%) trait hemoglobin E, enam (0.8%) hemoglobin E/talasemia-α dan lima (0.7%) trait talasemia-β. Hemoglobin Constant Spring dan hemoglobin A2 prime dilihat dalam dua (0.3%) kes; dan Hemoglobin Lepore serta varian rantaian alpha dalam satu (0.2%) kes. Walaubagaimanapun talasemia-α dan talasemia-β hemoglobin A2 normal tidak boleh diabaikan dalam 190 (26%) kes dan memerlukan analisa DNA untuk pengenalpastian jenis talasemia tersebut. \r\n            Saringan talasemia ke atas penderma darah adalah lebih mudah dan berkesan. Oleh yang demikian saringan populasi yang meluas termasuk penderma darah boleh memberi manfaat terhadap program saringan talasemia yang sedia ada di Malaysia. \r\n\r\n', 1),
(1095, 1087, 'xxx', 1),
(1098, 1090, NULL, NULL),
(1105, 1097, '<p>\r\n	Kajian ini bertujuan untuk mengenal pasti faktor-faktor yang berkait dengan penyakit ginjal kronik tahap 3b di kalangan pesakit kencing manis yang dirawat di Pusat Perubatan Primer UKM, khususnya peranan dos ubatan yang menyekat angiotensin. Kajian ini merupakan kajian kes-kawalan pilot yang tidak dipadankan dan dijalankan di sebuah pusat pengajaran perubatan primer. Data klinikal 25 kes pesakit dengan penyakit ginjal kronik tahap 3b (GFR 30-45ml/min/1.73m2) dalam tahun 2012 dipilih sebagai kes untuk kajian ini. Di samping itu, 103 pesakit diabetes yang mempunyai GFR lebih daripada 45ml/min/1.73m2 dalam tahun 2012 dipilih sebagai kawalan. Sampel dipilih secara rawak sistematik. Data pesakit diperolehi melalui rekod pesakit diabetes, sistem maklumat kesihatan komputer, dan fail perubatan pesakit. Analisis univariat menggunakan ujian chi square, t-test, Fisher&rsquo;s exact test dan Mann-Whitney U-test. Regresi logistik digunakan untuk mengenal pasti faktor-faktor yang berkait dengan pembentukan penyakit ginjal kronik tahap 3b. Kes dan kawalan berbeza dari segi umur, jangkamasa diabetes, penggunaan dan dos ubat penyekat angiotensin, tekanan darah sistolik dan GFR permulaan. Regresi logistic menunjukkan bahawa tekanan darah sistolik (Adjusted OR= 1.08, 95% CI= 1.02-1.14, p=0.013) and GFR tahap asas (Adjusted OR= 0.90, 95% CI= 0.85-0.95, p&lt;0.001) berkait secara signifikan dengan pembentukan penyakit ginjal kronik tahap 3b di kalangan pesakit diabetes. Penggunaan dos ubat penyekat angiotensin yang maksimum memperlihatkan kesan perlindungan (Adjusted OR= 0.14, 95% CI=0.85-0.95, p=0.025). Kajian ini menyokong keperluan kawalan tekanan darah sistolik yang baik untuk melambatkan kemerosotan fungsi ginjal. Dos ubat penyekat angiotensin perlu dioptimumkan di kalangan pesakit-pesakit ini.</p>\r\n', 1),
(1109, 1101, '<p>\r\n	Jangkitan Clostridium difficile (CDI) menyebabkan cirit-birit ringan hingga tenat dan kolitis pseudomembran di kalangan pesakit yang mempunyai pendedahan antibiotik terdahulu. Walaupun CDI berleluasa di seluruh dunia, data epidemiologi berkaitan CDI secara relatifnya adalah sedikit di Malaysia. Kajian ini bertujuan menentukan prevalen dan insiden CDI di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Spesimen tinja tidak berbentuk daripada 147 pesakit yang disyaki menghidap CDI dari 1 November 2011 hingga 31 Oktober 2012 telah digunakan dalam kajian ini. Kehadiran toksin A dan/atau B C. difficile dikesan dengan menggunakan kit imunokromatografi komersial (Wampole&trade; Tox A/B QuikChek). Data pengamatan dikumpul daripada rekod kesihatan pesakit bagi mendapatkan ciri-ciri demografi dan klinikal. Keseluruhan prevalen dan insiden CDI di PPUKM masing-masing adalah 6.1% dan 5.2 kes per 10 000 hari-pesakit. Di kalangan sembilan orang pesakit CDI, 77.8% adalah lelaki dan 55.6% berbangsa Cina. CDI paling kerap berlaku di wad perubatan (88.9%). Median umur adalah 60 tahun dan median tempoh inap di hospital adalah 13 hari. Majoriti (88.9%) pesakit CDI telah menerima antibiotik lapan minggu sebelum mendapat CDI. Penisilin-perencat-beta-laktamase merupakan antibiotik yang paling kerap digunakan. Lima (55.6%) pesakit CDI telah menerima ubatan penindas asid. Kadar kematian di hospital adalah 22.2%. Kesimpulannya, prevalen dan insiden CDI dalam institusi ini secara relatifnya adalah rendah dan berlaku secara sporadik.</p>\r\n', 1),
(1116, 1108, '<p>\r\n	Kebolehan membuat keputusan dalam rawatan merupakan salah satu kemahiran yang sangat penting untuk dimiliki oleh seseorang jururawat di dalam melaksanakan tugasannya. Tujuan kajian ini adalah untuk mengetahui persepsi di kalangan pelajar kejururawatan akan kebolehan mereka membuat keputusan. Satu kajian-rentas diskriptif menilai tahap persepsi dalam kalangan pelajar kejururawatan telah dijalankan di sebuah hospital tertiari dengan menggunakan versi modifikasi Skala Membuat Keputusan Dalam Kejururawatan (SMKDK) diadaptasi dari Jenkins (1985). Seramai 54 orang pelajar kejururawatan telah mengambil bahagian di dalam kajian ini yang mana skor purata SMKDK yang baik iaitu sebanyak 124.24&plusmn;12.713 telah direkodkan. Di dalam empat sub-skala SMKDK purata skor yang didapati adalah seperti berikut: mencari alternatif (33.24&plusmn;4.821), mendapatkan bantuan (28.74&plusmn;3.514), penilaian dan penilaian semula (31.43&plusmn;3.922), dan mencari maklumat (30.83&plusmn;4.765). Daripada bilangan pelajar tersebut hanya 19(35%) peserta telah memilih kejururawatan sebagai pilihan pertama. Juga didapati 37(69%) peserta berpuas hati dengan kompetensi kejururawatan mereka. Terdapat perbezaan yang signifikan di antara subskala mencari alternatif dan penilaian-penilaian-semula jika dibanding pada kumpulan yang memilih kejururawatan sebagai pilihan pertama (p&lt; 0.05). Juga terdapat perbezaan yang signifikan di antara mencari alternatif berbanding puas hati terhadap kompetensi kejururawatan (p&lt;0.05). Perbezaan yang signifikan juga ditunjukkan di antara tahap pendidikan berbanding mencari alternatif dan maklumat (p&lt;0.05). Pelajar kejuruwatan menunjukkan persepsi bahawa mereka berkebolehan membuat keputusan yang baik. Walaupun bidang kejururawatan ini bukan pilihan pertama bagi kebanyakan pelajar tetapi kebolehan mereka mencari alternatif dan melakukan penilaian serta menilai semula membolehkan mereka berjaya dalam membuat keputusan yang baik. Kepuasan dalam bidang yang diceburi juga membantu di dalam membuat keputusan yang baik di dalam bidang jaga-rawatan kritikal ini.</p>\r\n', 1),
(1120, 1112, '<p>\r\n	Akses untuk laluan intravena (IV) adalah amat penting didalam rawatan resusitasi pesakit kecemasan. Kerumitan mengakses laluan intravena akan menyebabkan kesukaran dalam pemberian cecair dan ubat yang boleh mempengaruhi kesan rawatan kepada pesakit. Dalam keadaan tiada laluan intravena, akses alternatif adalah kanulasi intraosseous. Pada masa ini, tidak ada data mengenai kelaziman dan corak pengetahuan tentang kanulasi intraosseous dikalangan paramedik kecemasan dalam penerbitan negara kita, walaupun penggunaan kanulasi intraosseous untuk pesakit kecemasan sedang diamalkan. Oleh itu, tujuan kajian ini adalah untuk mengenalpasti tahap pengetahuan tentang kanulasi intraosseous dikalangan paramedik kecemasan. Pengetahuan berkaitan dengan kanulasi intraosseous dikalangan paramedik kecemasan dinilai melalui soalan ujian berstruktur yang disahkan. Lima belas paramedik mengambil bahagian didalam kajian ini. Majoritinya adalah lelaki (86.6%). Kumpulan umur adalah diantara 22-45 tahun. Pengalaman bekerja pula adalah diantara 2-13 tahun. Terdapat hanya 1 daripada 15 peserta yang telah mencapai skor markah 75%. Majoriti (10) mencapai skor markah diantara 40% hingga 50%. Ini menunjukan bahawa pengajaran kanulasi intraosseous dikalangan paramedik kecemasan perlu diberi penekanan.</p>\r\n', 1),
(1122, 1114, '<p>\r\n	Sistem genitourinari ialah tapak kedua paling lazim untuk tuberkulosis (TB) selepas sistem pulmonary. Tuberkulosis prostat ialah manifestasi tuberculosis yang jarang berlaku dalam sistem genitourinari. Ia lebih jarang berlaku pada individu yang mempunyai tahap imun yang memuaskan. TB prostat selalunya ditemui semasa reseksi transuretral prostat (TURP). Kami melaporkan kes prostat TB dalam lelaki berusia 70 tahun yang menghidap hyperplasia prostat benigna selama dua tahun. TB didiagnosa secara kebetulan selepas TURP. Dalam laporan kes ini, kami juga akan membincangkan ujian-ujian lain yang boleh dijalankan untuk mendiagnosis tuberculosis dalam sistem genitourinari.</p>\r\n', 1),
(1124, 1116, '<p>\r\n	Sellulitis orbital merupakan salah satu daripada keadaan yang boleh mengancam nyawa dan tidak seharusnya terlepas pandang. Ianya harus dibezakan dengan sellulitis praseptal. Ini terjadi akibat proses jangkitan yang melibatkan struktur sekitar mata posterior kepada septum orbital. Indeks kecurigaan yang tinggi harus ada bagi mendiagnosa keadaan ini. Kami membentangkan satu kes melibatkan seorang gadis berumur 15 tahun yang datang disebabkan oleh bengkak pada dahi sebelah kanan yang membesar dengan cepat. Beliau juga mengalami demam tinggi, sakit kepala dan nanah pada bengkakan tersebut. Beliau didiagnosa mengalami sepsis yang disebabkan abses dahi dan diberi rawatan dengan antibiotik serta di masukkan ke wad. Walau bagaimanapun, keadaan beliau bertambah tenat dan terpaksa dimasukkan ke unit rawatan rapi. Hasil penyiasatan menunjukan terdapat koleksi cecair pada ruang retrobulbar dan pembedahan segera dilakukan bagi mengeluarkan nanah. Namun, keadaan beliau bertambah teruk dan akhirnya beliau meninggal pada hari ke-10.</p>\r\n', 1),
(1129, 1121, '<p>\r\n	Eosinophilic gastroenteritis, penyakit inflamasi yang tidak diketahui puncanya, biasanya melibatkan infiltrasi eosinophilic ke dalam perut dan usus kecil. Di sini, kami menerangkan kes gastroenteritis eosinophilic yang jarang berlaku yang melibatkan keseluruhan saluran penghadaman sebagai manifestasi sindrom hypereosinophilic. Seorang wanita berusia 22 tahun datang dengan cirit-birit, lelehan pleural, ascites dan eosinophil darah periferi yang tinggi. Spesimen najis adalah negatif untuk parasit, ova, bakteria dan kulat. Kajian endoskopi menunjukkan pangastritis dan duodenitis. Spesimen biopsi esofagus, perut, duodenum, usus kecil, dan kolon menunjukkan penyusupan eosinophilic. Diagnosa sindrom hypereosinophilic dengan gastroenteritis eosinophilic melibatkan saluran penghadaman keseluruhan telah dibuat. Oleh itu, pesakit telah dirawat dengan prednisolone. Gejala dan eosinophilia periferal sembuh dengan rawatan, dan penyiasatan radiologi menunjukkan lelehan pleura hilang selepas rawatan. Kes ini menggambarkan spektrum penyakit ini, di mana ia melibatkan saluran pencernaan keseluruhan dan ia juga menekankan kepentingan diagnostik biopsi endoskopi.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(1133, 1125, '<p>\r\n	Variasi hepatic biasa atau kolik arteri tengah boleh menyumbang kepada komplikasi yang timbul ketika pembedahan hati dan pembedahan abdomen bahagian atas. Kes corak bercabang ganjil pada cabang ventral aorta abdominal pada mayat lelaki berusia 50 tahun dilaporkan. Batang seliak berasal daripada aspek ventral bagi aorta abdominal dan bercabang kepada gastrik kiri dan arteri limpa. Arteri hepatic umum berasal secara langsung dari abdominal aorta, 0.2 cm di bawah asal batang seliak. Arteri kolik tengah berasal daripada segmen proksimal bagi arteri limpa, bukannya daripada arteri mesenterik unggul. Satu cabang berasingan dari kolik pertengahan mesenterik arteri unggul tidak dilihat. Variasi anatomi dalam corak bercabang batang seliak dan arteri mesenterik unggul tidak kerap, bagaimanapun asal-usul variasi seiring cawangan batang seliak dan arteri kolik pertengahan adalah anomali yang jarang berlaku dan oleh itu, pengetahuan mengenainya akan membantu dalam prosedur pembedahan abdomen dan imej berpandukan intervasi.</p>\r\n', 1),
(1139, 1131, 'Tujuan kajian ini adalah untuk menyiasat kelaziman hilang kawalan kencing selepas bersalin (PNUI), faktor-faktor risiko yang berkaitan dengannya dan penglibatan dalam mengamalkan penjagaan selepas bersalin mengikut tradisi Melayu (TMCC) di Kelantan, Malaysia. Satu kajian keratan rentas telah dijalankan di kalangan 362 wanita selepas bersalin yang berumur antara 18 - 45 tahun dan yang antara tiga hingga lima bulan selepas bersalin. Para peserta telah dipilih melalui persampelan mudah di enam Klinik Kesihatan Ibu dan Anak yang terletak di daerah yang agak sama. Analisis deskriptif dan inferensi termasuk, analisis faktor, Pearson khi-kuasa dua, ujian Mann-Whitney U dan analisis terurus logistik telah digunakan untuk menganalisis data. Secara keseluruhan, PNUI dilaporkan oleh 80 wanita (22.1%). Cara bersalin melalui faraj dikaitkan dengan PNUI. Semua wanita selepas bersalin dalam konteks kajian didapati mengamalkan TMCC. Kajian ini juga menunjukkan bahawa wanita yang mempunyai PNUI lebih cenderung untuk melibatkan diri dalam komponen ''penjagaan Badan’ TMCC. Hilang kawalan kencing adalah lazim di kalangan wanita selepas bersalin di Kelantan, Malaysia. Penyelidikan selanjutnya terhadap kesan komponen ''penjagaan badan'' TMCC pada PNUI perlu dilakukan. ', 1),
(1145, 1137, '<p>\r\n	Midazolam adalah salah satu ubat yang paling biasa digunakan untuk ubat pelali dalam Jabatan Kecemasan. Ini adalah satu kajian retrospektif yang dijalankan ke atas 380 pesakit dari Disember 2012 hingga Mei 2014 di Jabatan Kecemasan (ED) PPUKM. Objektifnya adalah untuk mendapatkan kekerapan kesan sampingan dan korelasi kepada pelbagai faktor iaitu sosio-demografi, kumpulan umur dan penyakit-penyakit utama. KEPUTUSAN: Daripada 380 pesakit, 35 pesakit mempunyai kesan sampingan (20 pesakit dengan midazolam sahaja, 15 pesakit dengan kombinasi ubat-ubatan) Umur purata adalah 42 tahun dan dos purata midazolam adalah 3.5mg. Ubat lain yang paling biasa adalah gabungan fentanyl. Kadar komplikasi keseluruhan midazolam adalah 5.3%. Kesan sampingan yang paling biasa yang dicatatkan adalah sifat tidur berlebihan (1.6%). Kesan sampingan yang lain adalah reaksi allergi kulit (1.1%), muntah (0.8%), sakit kepala (0.8%) dan tekanan darah rendah (0.5%). Tidak ada hubungan yang signifikan antara faktor-faktor sosio-demografi dan kombinasi ubat-ubatan dengan kesan sampingan midazolam pada pesakit. Kesimpulan daripada kajian ini ialah midazolam adalah dadah yang sangat selamat kerana tiada kesan sampingan yang mengancam nyawa dijumpai. Terdapat kemungkinan bahawa kesan-kesan sampingan direkodkan boleh disebabkan oleh faktor-faktor lain seperti kecederaan atau penyakit dan kombinasi dengan ubat-ubatan lain.</p>\r\n', 1),
(1152, 1144, 'Sepsis menyebabkan kadar kematian dan morbiditi pesakit di hospital yang tinggi. Kepekatan laktat statik dan kadar pengurangan paras laktat awal dicadangkan sebagai faktor utama penentuan kadar kematian pesakit sepsis. Kajian ini mengaplikasikan kepekatan laktat dan kadar pengurangan paras laktat pada tempoh 6 jam pertama di Jabatan Kecemasan untuk meramalkan kadar kematian pesakit sepsis selepas 28 hari. Pesakit yang menghidapi sepsis , sepsis teruk dan renjatan sepsis yang menerima rawatan di jabatan kecemasan Universiti Kebangsaan Malaysia akan dimasukkan dalam kajian ini. Kepekatan laktat static darah pesakit pada masa permulaan kemasukan pesakit, pada 1 jam dan pada 6 jam akan dikaji. Pesakit kemudiannya diberi rawatan yang berpatutan mengikut diagnosis sepsis, iaitu rawatan piawai atau rawatan awal berasalkan sasaran untuk sepsis. Selepas 28 hari, pesakit akan dihubungi melalui telefon, emel atau laporan perubatan pesakit. Kemudian, pesakit akan dikatogorikan kepada golongan yang hidup atau mati. Dalam kajian ini, kepekatan laktat menujukan hubung kait yang erat dengan golongan yang mati berbanding dengan golongan yang hidup dalam ketiga-tigaan masa, iaitu jam 0, 1 dan 6 (p<0.05). Akan tetapi, kadar pengurangan paras laktat tidak menunjukkan sebarang hubung kait dengan kadar kematian pesakit selepas 28 hari. Kesimpulannya, kepekatan laktat static merupakan petunjuk yang baik untuk meramalkan kadar kematian sepsis berbanding dengan kadar pengurangan paras laktat. Walaupun kadar pengurangan paras laktat tidak dapat membuktikan sebarang hubung kait dengan kadar kematian selepas 28 hari, kajian kami menunjukkan ia masih boleh dimanfaatkan sebagai pemantau yang baik semasa merawat pesakit sepsis.\r\nKata Kunci: Sepsis, Kepekatan Laktat Statik, Kadar Pengurangan Paras Laktat Awal, Kadar Kematian\r\n', 1),
(1154, 1146, NULL, NULL),
(1157, 1149, 'Laporan ini mengetengahkan peranan Perawatan komuniti asertif (ACT) dalam merawat sebuah keluarga yang kebanyakan ahlinya menghidap penyakit mental; skizofrenia. Kami melaporkan satu kes iaitu seorang wanita dalam usia pertengahan yang menghidapi penyakit skizofrenia resistan pada rawatan (TRS) yang tinggal bersama dua lagi ahli keluarganya yang menghidapi penyakit yang sama. Mereka dijaga oleh seorang penjaga yang menghidapi kemurungan major yang terkesan dari bebanan menanggung ahli keluarga yang sakit mental. ACT melalui pendekatannya yang holistik, terbukti dapat membantu mengurangkan risiko kemasukan ke hospital serta dapat membantu dalam penambahbaikan kawalan penyakit dan kualiti hidup keluarga ini.\r\n\r\n\r\n\r\n', 1),
(1160, 1152, 'Lavaj Orogastrik telah dijalankan sejak 200 tahun yang lalu untuk pesakit yang telah diracun atau meracuni diri sendiri. Kekerapan procedur ini telah menurun sedekad yang lalu disebabkan factor risiko yang mengatasi manfaat. Seorang gadis remaja yang telah menelan sebotol racun perosak dalam tempoh masa yang sebelum pengosongan gastrik telah tiba di jabatan kecemasan kami. Pesakit itu diresusitasikan dan lavaj orogastrik telah dilakukan. 50 cc bahan toksik telah disedut dan penawar toksin itu telah diberi. Pesakit kemudian telah dimasukkan ke Unit Rawatan Rapi dan seterusnya dibenarkan pulang dari hospital 5 hari kemudian. Pengurusan saluran pernafasan yang betul , teknik yang betul , pemilihan yang betul bagi pesakit dan pemantauan yang mencukupi adalah penting bagi kejayaan lavaj orogastrik .\r\n', 1),
(1162, 1154, '<p>\r\n	Papillary Nasopharyngeal Adenocarcinoma Gred Rendah (LGPAC) adalah sejenis tumor berasal dari mukosa yang jarang berlaku berbanding kanser nasofarinks jenis &ldquo;keratinized&rdquo;. Secara histology, terdapat diferensiasi jenis papillary yang juga boleh didapati dalam kes metastatik karsinoma papilari tiroid. Keadaan ini menyebabkan cabaran bagi pihak patologi untuk membezakan antara kes primari atau metastatik. Thyroid Transcription Factor-1 (TTF-1) diekspreskan dalam subset Papillary Nasopharyngeal Adenocarcinoma dan ini merupakan alat diagnostic yang berguna. LGPAC adalah tumour malignan bersifat benign. Diagnosis awal beserta pembedahan membuang keseluruhan tumour melalui cara konvensional atau endoskopik boleh memberikan prognosis bagus dan risiko berlakunya lagi tumour ini adalah rendah.</p>\r\n', 1),
(1166, 1158, '<p>\r\n	Osteoporosis dikenali sebagai penyakit senyap kerana tidak mempunyai tanda-tanda awal. Ini disebabkan oleh kepadatan tulang yang berkurangan secara perlahan-lahan seiring dengan peningkatan usia. Insiden penyakit ini semakin meningkat setiap tahun di seluruh dunia. Mengukur ketumpatan mineral tulang (BMD) menggunakan densitometry tulang konvensional (DXA) adalah praktikal dalam diagnosis osteoporosis tetapi kosnya adalah tinggi dan tidak dapat dilaksanakan dalam masyarakat. Untuk mengukur ketumpatan tulang, &ldquo;quantitative ultrasound&rdquo; (QUS) adalah teknik yang agak moden untuk diagnosis osteoporosis. Ianya agak mudah, konsisten, lebih murah dan kaedah yang selamat berbanding dengan teknik densitometry yang lain. Kedua-dua parameter QUS yang diukur pada masa kini adalah ultrasound jalur pengecilan (BUA) dan kelajuan bunyi (SOS). QUS juga dapat menjangka risiko patah. Ianya kini digunakan untuk memantau tindakbalas kepada rawatan anti-osteoporosis. Kajian in-vitro menunjukkan bahawa indeks QUS berhubungkait dengan BMD, bentuk tulang mikro dan parameter mekanikal. Oleh yang demikian, QUS berupaya untuk menjadi teknik baru untuk penilaian tulang.</p>\r\n', 1),
(1172, 1164, '<p>\r\n	Penyakit kekurangan glucose-6-phosphate-dehyrogenase (G6PD) merupakan penyakit kekurangan enzim paling lazim di dunia termasuklah Malaysia. Saringan kekurangan separa enzim ini menggunakan darah tali pusat adalah sangat penting memandangkan mereka juga berisiko untuk mendapat hemolisis akut. Dalam kajian ini, kadar prevalens bagi kekurangan separa G6PD ditentukan dengan menggunakan kit ujian OSMMR-D dimana ianya mengintegrasikan kaedah pernormalan hemoglobin dan kadarnya dibandingkan dengan kaedah ujian titik pendafluoran (FST). Sejumlah 236 subjek dari kalangan kanak-kanak, berumur di antara 1 bulan hingga 12 tahun dan; 614 bayi-bayi perempuan telah dipilih untuk kajian ini. Penentuan julat normal aktiviti G6PD dan; paras penentu untuk kekurangan separa dan sepenuhnya ditentukan mengikut WHO Working Group (1989). Kadar prevalens untuk kekurangan separa untuk kedua-dua kumpulan pesakit (perempuan) dibuat menggunakan kit esei enzim ini dan keputusannya dibandingkan dengan FST. Dalam kajian ini, didapati 15.7% (18/115) kanak-kanak perempuan diklasifikasikan kekurangan separa G6PD menerusi kaedah ujian esei enzim (aktiviti G6PD: 4.23-5.26U/gHb), tetapi FST hanya dapat mengesan 0.9% (1/115) yang mempunyai aktiviti G6PD minimal. Kadar prevalens kekurangan separa G6PD pada bayi-bayi perempuan adalah 3.42% (21/614) menggunakan ujian esei enzim berbanding 0.49% (3/614) menggunakan FST. Kesimpulannya, hasil kajian ini membutikan bahawa ujian FST gagal mengesan pesakit kekurangan separa G6PD (heterozigot). Sehubungan dengan itu, kami menyarankan penggunaanujian kuantitatif esei G6PD dengan OSMMR-D kit memandangkan ianya lebih sensitif untuk mengesan penyakit kekurangan G6PD dalam bayi-bayi perempuan berbanding dengan FST.</p>\r\n', 1),
(1177, 1169, '<p>\r\n	Tujuan kajian ini adalah untuk menyiasat kelaziman hilang kawalan kencing selepas bersalin (PNUI), faktor-faktor risiko yang berkaitan dengannya dan penglibatan dalam mengamalkan penjagaan selepas bersalin mengikut tradisi Melayu (TMCC) di Kelantan, Malaysia. Satu kajian keratan rentas telah dijalankan di kalangan 362 wanita selepas bersalin yang berumur antara 18-45 tahun dan antara tiga hingga lima bulan selepas bersalin. Para peserta telah dipilih melalui persampelan mudah di enam Klinik Kesihatan Ibu dan Anak yang terletak di daerah yang agak sama. Analisis deskriptif dan inferensi termasuk, analisis faktor, Pearson khi-kuasa dua, ujian Mann-Whitney U dan analisis terurus logistik telah digunakan untuk menganalisis data. Secara keseluruhan, PNUI dilaporkan oleh 80 wanita (22.1%). Cara bersalin melalui faraj dikaitkan dengan PNUI. Semua wanita selepas bersalin dalam konteks kajian didapati mengamalkan TMCC. Kajian ini juga menunjukkan bahawa wanita yang mempunyai PNUI lebih cenderung untuk melibatkan diri dalam komponen &lsquo;Penjagaan Badan&rsquo; TMCC. Hilang kawalan kencing adalah lazim di kalangan wanita selepas bersalin di Kelantan, Malaysia. Penyelidikan selanjutnya terhadap kesan komponen &lsquo;penjagaan badan&rsquo; TMCC pada PNUI perlu dilakukan.</p>\r\n', 1),
(1182, 1174, '<p>\r\n	Tujuan penyelidikan ini dijalankan ialah untuk menilai tahap pengetahuan masyarakat terhadap faktor risiko, tanda-tanda, rawatan dan sumber maklumat berhubung strok dan serangan iskemia transien, dan seterusnya menjana profil risiko strok and melihat hubungkaitnya dengan pengetahuan mereka. Menggunakan borang kajiselidik yang telah divalidasi dan dicuba, 112 orang telah berjaya disoalselidik pada dua acara kemasyarakatan yang berbeza iaitu Hari Kesedaran Strok dan Hari Terbuka Kemasyarakatan. Orang ramai telah mengisi borang kajiselidik dan kemudian menjalani ujian saringan kesihatan. Seratus orang telah layak untuk kajian ini. Maklumat yang dikumpul telah dianalisa menggunakan SPSS versi 20. Secara keseluruhannya, pengetahuan awam didapati sangat lemah. Hanya 35% daripada peserta dikategorikan sebagai mempunyai pengetahuan yang memuaskan berkaitan tanda-tanda strok (p=0.94) dan 29% mempunyai pengetahuan memuaskan berkaitan faktor risiko (p=0.46). Setelah dibahagikan antara kumpulan risiko, didapati peratusan pengetahuan mengenai strok yang memuaskan adalah: pesakit berisiko tinggi (41%), berisiko sederhana (30%) dan berisiko rendah (26%). Tiada seorang peserta pun yang tahu berkenaan wujudnya rawatan bagi serangan strok akut atau kepentingan rehabilitasi sebagai sebahagian daripada pengurusan strok. Kesedaran awam berhubung faktor risiko, tanda-tanda dan pengurusan strok adalah pada tahap yang membimbangkan.</p>\r\n', 1),
(1194, 1186, '<div>\r\n	Program saringan talasemia telah diwujudkan di Malaysia semenjak tahun 2004. Program ini bertujuan untuk mengurangkan beban akibat penyakit ini dengan mengenal-pasti pembawa talasemia. Walaubagaimanapun, sambutan terhadap aktiviti saringan ini didapati kurang memuaskan kerana kurangnya kesedaran orang ramai akan kepentingan saringan talasemia. Kaedah alternatif adalah menyaring penderma darah. Matlamat kajian ini adalah untuk mengetahui prevalens pembawa talasemia di kalangan penderma darah yang sihat. Tujuh ratus tiga puluh lapan penderma darah yang sihat telah disaring di Hospital Tengku Ampuan Rahimah, Klang menggunakan kaedah cation-exchange high performance liquid chromatography (HPLC) dari Julai hingga September 2010. Semua kes varian hemoglobin seterusnya diproses menggunakan gel electrophoresis pada pH alkali.</div>\r\n<div>\r\n	Hasil kajian menunjukkan penderma darah terdiri daripada bangsa Melayu 413 (56%), India 162 (22%), Cina 148 (20%) and lain-lain 15 (2%). Terdapat 19 (2.6%) trait hemoglobin E, enam (0.8%) hemoglobin E/talasemia-&alpha;&alpha; dan lima (0.7%) trait talasemia-&beta;. Hemoglobin Constant Spring dan hemoglobin A2 prime dilihat dalam dua (0.3%) kes; dan Hemoglobin Lepore serta varian rantaian alpha dalam satu (0.2%) kes. Walaubagaimanapun talasemia-&alpha;&alpha; dan talasemia-&beta; hemoglobin A2 normal tidak boleh diabaikan dalam 190 (26%) kes dan memerlukan analisa DNA untuk pengenalpastian jenis talasemia tersebut. Saringan talasemia ke ataspenderma darah adalah lebih mudah dan berkesan. Oleh yang demikian, saringan populasi yang meluas termasuk penderma darah boleh memberi faedah kepada program saringan talasemia yang sedia ada di Malaysia.</div>\r\n', 1),
(1200, 1192, '<p>\r\n	Disfungsi seksual di kalangan pesakit kanser ginekologi adalah prevalens. Pada kebanyakan situasi, disfungsi seksual di kalangan penderita kanser ginekologi adalah di bawah paras pengenalpastian di mana terdapat pelbagai faktor morbiditi psikologi, faktor berpelbagaian yang berinteraktif berserta kengganan pesakit menceritakan masalah mereka kepada doktor perawat. Menentukan faktor berisiko disfungsi seksual di kalangan pesakit akan membuka ruang untuk kita memberi lebih perhatian kepada golongan yang memerlukan perhatian serta membolehkan satu strategi untuk mengenalpasti disfungsi seksual di peringkat awal, agar usaha pencegahan dan rawatan boleh dilaksanakan. Kajian ini bertujuan untuk menentukan prevalens dan faktor berisiko disfungsi seksual di kalangan pesakit kanser ginekologi di Hospital Sultanah Bahiyah, Alor Star. Fungsi seksual dikalangan 83 penderita kanser ginekologi yang berkahwin dinilai dengan skala swa-penilaian MVFSFI (Malay version Female Sexual Function Index). Skala swa-penilaian WHOQOL-BREF (World Health Organization-Quality of Life-26) yang digunakan untuk menilai domain kualiti kehidupan sementara MINI (Mini International Neuropsychiatry Interview) pula untuk menilai masalah psikiatri di kalangan pesakit. Kadar prevalens disfungsi seksual di kalangan pesakit kanser ginekologi ialah 65% (54/83). Disfungsi seksual berkait secara signifikan dengan tahap pendidikan yang rendah (kadar odds, OR: 3.055; sela keyakinan, CI 1.009-9.250), tempoh penderitaan kanser yang pendek (OR 0.966, CI 0.966- 0.998), rawatan kemoterapi yang berterusan (OR 3.045, CI 1.149-8.067), persepsi tentang kesakitan (OR 3.230, CI 1.257-8.303), kurangnya perhubungan seks (OR 1.862) dan tiga domain kualiti kehidupan termasuk kesihatan fizikal, psikologi dan perhubungan sosial (masing-masing OR 0.942, CI 0.908-0.978; OR 0.955, CI 0.916-0.995; OR 0.933, CI 0.894-0.973). Walaubagaimana pun, disfungsi seksual tidak berkait dengan kemurungan yang dialami oleh penderita kanser ginekologi (&chi;2 = 1.224, p = 0.268). Kesimpulannya, prevalens disfungsi seksual di kalanganpenghidap kanser ginekologi adalah tinggi, sama seperti kajian-kajian lain yang telah dijalankan. Memandangkan prevalens disfungsi seksual adalah berpelbagaian dimensi, justeru proses penilaian dan perawatan seharusnya mendukung konsep holistik serta berorientasikan pesakit.</p>\r\n', 1),
(1202, 1194, '<p>\r\n	Kajian ini bermatlamat meninjau faktor-faktor sosio-demografi yang mempunyai impak pada merokok and pengunaan arak di kalangan orang dewasa di Pulau Pinang (Malaysia). Data tinjau selidik keratan rentas yang mengandungi 398 responden telah digunakan untuk menjalankan analisis. Kajian ini menggunakan model regresi logistik untuk memeriksa faktor-faktor yang mempengaruhi kemungkinan merokok dan pengunaan arak. Keputusan menunjukkan jantina, kaum dan pendidikan dapat mempengaruhi merokok. Lelaki (OR: 26.678) menunjukkan kemungkinan yang lebih untuk merokok berbanding dengan wanita. Manakala, kaum Cina (OR: 0.177), kaum India/lain-lain (OR: 0.331) dan individu yang berpendidikan tinggi (OR: 0.258) menunjukkan kemungkinan yang kurang untuk merokok berbanding dengan yang lain. Dari segi pengunaan arak, jantina, kaum dan taraf perkahwinan dapat mempengaruhi kemungkinan pengunaan arak. Lelaki (OR: 5.051), kaum Cina (OR: 37.796) dan kaum India/lain-lain (OR: 10.863) menunjukkan kemungkinan yang lebih untuk menggunakan arak berbanding dengan lain. Manakala, individu yang berkahwin (OR: 0.380) menunjukkan kemungkinan yang kurang untuk menggunakan arak berbanding dengan individual yang belum berkahwin. Berpandukan penemuan kajian ini, pelbagai tindakan pencegahan telah dicadangkan.</p>\r\n', 1),
(1205, 1197, '<p>\r\n	Palsi serebrum (CP) adalah penyebab biasa kecacatan kanak-kanak. Ia mempunyai kesan yang besar kepada ibu bapa dan penjaga, terutama apabila ianya melibatkan lebih daripada satu ahli keluarga yang sama. Walaupun CP kekeluargaan (familial CP) sangat jarang berlaku, kes-kes yang dilaporkan di kalangan kanak-kanak dari perkahwinan yang mempunyai pertalian darah atau sebaliknya, dan kehamilan kembar telah mencadangkan kemungkinan ianya melibatkan masalah keturunan genetik yang kompleks. Pakar perubatan perlu sedar tentang CP kekeluargaan untuk pengesanan dan kaunseling awal. Kami melaporkan kes CP yang jarang berlaku pada dua adik-beradik lelaki daripada perkahwinan yang tiada mempunyai pertalian kekeluargaan.</p>\r\n', 1),
(1211, 1203, '<p>\r\n	Ulser trofik telah muncul sebagai satu daripada komplikasi utama diabetis mellitus dan penyakit Hansen. Dalam kes bersiri ini, kesan penyembuhan ulser trofik menggunakan &lsquo;but plaster bersentuhan penuh&rsquo; yang diperbuat daripada plaster Paris dikaji ke atas 10 subjek. Seramai lima subjek adalah pesakit diabetis mellitus manakala lima subjek lagi adalah pesakit penyakit Hansen. Saiz luka ulser sebelum dan selepas rawatan &lsquo;but plaster bersentuhan penuh&rsquo; diukur sebagai hasil kajian. Kesemua 10 subjek menunjukkan pengurangan saiz luka selepas 15 hari penggunaan &lsquo;but plaster bersentuhan penuh&rsquo;. Tiada kesan mudarat dikaitkan dengan rawatan ini. Pesakit dengan ulser trofik boleh menerima faedah daripada rawatan &lsquo;but plaster bersentuhan penuh&rsquo;.</p>\r\n', 1),
(1215, 1207, '<p>\r\n	Pembesaran payudara menggunakan implan silikon adalah prosedur estetik yang popular dengan profil keselamatan yang tinggi. Jangkitan implan silikon payudara adalah entiti klinikal yang jarang berlaku. Rawatan biasanya melibatkan pembuangan implan dan penggunaaan antibiotik. Laporan ini mengetengahkan satu kes jangkitan implan payudara yang berlaku hampir 15 tahun selepas pembedahan pertama.</p>\r\n', 1),
(1221, 1213, '<p>\r\n	Kepercayaan pesakit terhadap pengurursan kesakitan boleh menjejaskan hasil pengurusan kesakitan dan kualiti hidup. Tujuan kajian ini dijalankan adalah untuk menentukan kesan pakej pendidikan pra operasi sakit ke arah kepercayaan kesakitan antara pesakit-pesakit yang menjalani pembedahan ortopedik di hospital tertiari. Reka bentuk kajian &lsquo;pre-test pos test&rsquo; satu-kumpulan dijalankan pada pesakit-pesakit untuk menentukan impak pakej pendidikan tentang kesakitan terhadap skor &lsquo;post-test&rsquo; kepercayaan tentang kesakitan dikalangan pesakit yang menjalani pembedahan ortopedik. Tiga puluh responden telah diambil menggunakan &lsquo;convenience sampling&rsquo;. Pendidikan tentang kesakitan sebelum pembedahan berkaitan dengan pengurusan kesakitan diberikan kepada respons. Skor kepercayaan pra operasi dan postoperative sakit, pengurusan markah dan kesan-kesan sampingan telah diukur dengan menggunakan borang soal-selidik halangan (BQ-13). Keputusan melaporkan perbezaan yang signifikan antara skor &lsquo;pre-test&rsquo; (Mean = 41.87, Standard Deviation = 11.467) dan &lsquo;post-test&rsquo; (Mean = 34,80, Standard Deviation = 13.026) kepercayaan terhadap kesakitan (t = 2.84, p =0.004). Terdapat juga perbezaan yang signifikan antara skor &lsquo;pre-test&rsquo; (Mean = 37.10, Standard Deviation = 10.610) dan skor &lsquo;post-test&rsquo; (Mean = 30.80, Standard Deviation = 11.424), pengurusan kesakitan (t = 3.856, p =0.0005). Responden dan jantina (t =-2.403, p =0.023) dan bangsa (F = 5.038, p =0.014) melaporkan perbezaan yang signifikan iaitu p &lt; 0.05. Bagaimanapun, tidak terdapat perbezaan yang signifikan antara tahap pendidikan, etnik, sejarah pembedahan terlebih dahulu dengan kepercayaan kesakitan dengan p &gt; 0.05. Kesimpulannya, terdapat kesan positif daripada pakej pendidikan kesihatan berkenaan kepercayaan kesakitan dan pengurusan kesakitan responden yang telah menjalani pembedahan ortopedik di hospital tertiari. Pengukuhan program pendidikan kesakitan adalah penting untuk bagi mencapai matlamat pengurusan kesakitan postoperative yang optimum.</p>\r\n', 1),
(1227, 1219, '<p>\r\n	-</p>\r\n', 1),
(1228, 1220, '<p>\r\n	-</p>\r\n', 1),
(1231, 1223, '<p>\r\n	Takuk interkondil adalah tapak anatomi yang menarik kerana ia terbenam di ligament krusiat anterior. Objektif kajian ini ialah untuk mengkaji morfologi dan morfometri takuk interkondil femur pada tulang kering cadaver dengan penekanan pada implikasi klinikalnya. Kajian dilakukan ke atas 97 femur kering. Parameter yang diukur adalah lebar takuk interkondil, kedalaman takuk interkondil, lebar kondil dan kedalaman kondil. Bentuk interkondil juga dianalisa. Ukuran dibandingkan secara statistic merujuk kepada sebelah kanan dan sisi dan dibuat dalam bentuk jadual. Pemerhatian mendapati secara morfologi takuk interkondil adalah berbentuk &lsquo;U&rsquo; terbalik pada 71 (73.2%) specimen dan berbentuk &lsquo;V&rsquo; terbalik pada 26 (26.8%) spesimen. Min lebar takuk interkondil, kedalaman takuk interkondil, lebar kondil dan kedalamanan kondil adalah 11.9 &plusmn; 2.7 mm, 26.3 &plusmn; 2.4 mm, 72.9 &plusmn; 5.3 mm dan 57.3 &plusmn; 4.3 mm masing-masing. Pemerhatian mendapati tiada perbezaan statistik yang signifikan (p &gt; 0.05) antara sebelah kanan dan kiri. Indeks lebar dan kedalamanan lekuk didapati sebagai 0.25 dan 0.46 masing-masing. Data morfometri kajian ini adalah penting kepada pakar ortopedik bagi menghalang dan menguruskan kes kecederaan lutut. Kami percaya kajian ini telah menambah maklumat atas subjek berkaitan dan data ini boleh digunakan oleh pakar klinikal yang terlibat dalam membuat diagnosis dan mengurus masalah lutut.</p>\r\n', 1),
(1234, 1226, '<p>\r\n	Pesakit Penghalang Pulmonari Kronik (PPK) dilaporkan mengalami fungsi kardiovaskular tidak stabil yang membataskan aktiviti dan menjejaskan kualiti hidup. Latihan fizikal telah dibuktikan bertindak sebagai salah satu moderator ke atas batasan aktiviti tersebut.Bagaimanapun, protokal dan durasi latihan fizikal masih belum difahami sepenuhnya. Kajian ini menyiasat kesan latihan fizikal ke atas tekanan darah, kadar dengupan jantung dan Hasil Darab Kadar Tekanan bagi pesakit PPK. Sejumlah tiga puluh pesakit PPK yang berumur antara 40 hingga 55 tahun direkrut untuk kajian berdasarkan kriteria serta dan singkir dan dibahagikan kepada kumpulan eksperimen (15 pesakit) dan kumpulan kawalan (15 pesakit). Min beza tekanan darah antara bacaan sebelum dan selepas ujian dianalisa menggunakan ujian-t. Terdapat perbezaan signifikan bagi semua parameter antara kumpulan eksperimen dan kumpulan kawalan dengan p&lt;0.05. Tekanan darah diastolic adalah kurang signifikan berbanding tekanan darah sistolik. Ini menunjukkan bahawa aktiviti fizikal yang direka khas meningkatkan tahap kecergasan kardiovaskular pesakit PPK.</p>\r\n', 1),
(1241, 1233, '<p>\r\n	Kajian ini adalah bertujuan untuk mengetahui tahap kemurungan, keresahan dan tekanan dikalangan pelajar perubatan tahun klinikal Universiti Kebangsaan Malaysia (UKM) dan perlakuan mencari bantuan berdasarkan aspek psikologikal para pelajar. Satu kajian hirisan telah diadakan di Pusat Perubatan UKM selama tiga bulan. Responden kajian adalah terdiri dari para pelajar tahun tiga, empat dan lima melalui sampel sebanding. Pelajar diberikan borang soal selidik, yang mengandungi demografik sosial, &lsquo;Depression, Anxiety and Stress Scale 21&rsquo; versi Bahasa Malaysia (BM-DASS 21) dan juga soalan mengenai tabiat meminta pertolongan. Penilaian psikometrik menganggarkan tahap kemurungan, keresahan dan tekanan di kalangan prasiswazah. Manakala soalan mengenai tabiat meminta pertolongan menilai kegunaan fasiliti perubatan dan sumber bantuan yang lain. Sejumlah 350 para pelajar telah mengambil bahagian didalam kajian ini. Seramai 1.3% dari mereka dikesan berkemungkinan menghadapi kemurungan, 2.4% mengalami keresahan dan 2.4% merasa tertekan. Majoriti pelajar lebih suka meminta bantuan daripada rakan (283;74.5%) diikuti dengan ibubapa (275; 72.4%). Terdapat hubungkait yang signifikan di antara para pelajar yang mengalami keresahan dengan tabiat meminta bantuan daripada sumber yang formal seperti kaunselor (p=0.001) dan pegawai agama (p=0.03). Kesimpulannya, ia adalah penting untuk meningkatkan tahap kesedaran para pelajar berkenaan gangguan psikologi dan program saringan yang efisien patut diguna pakai untuk mengenalpasti pelajar yang berkemungkinan mengalami gangguan psikologi.</p>\r\n', 1),
(1248, 1240, '<p>\r\n	Sepsis menyebabkan kadar kematian dan morbiditi pesakit di hospital yang tinggi. Kepekatan laktat statik dan kadar pengurangan paras laktat awal dicadangkan sebagai faktor utama penentuan kadar kematian pesakit sepsis. Kajian ini mengaplikasikan kepekatan laktat dan kadar pengurangan paras laktat pada tempoh enam jam pertama di Jabatan Kecemasan untuk meramalkan kadar kematian pesakit sepsis selepas 28 hari. Pesakit yang menghidapi sepsis, sepsis teruk dan renjatan sepsis yang menerima rawatan di jabatan kecemasan Universiti Kebangsaan Malaysia akan dimasukkan dalam kajian ini. Kepekatan laktat static darah pesakit pada masa permulaan kemasukan pesakit, pada satu jam dan pada enam jam akan dikaji. Pesakit kemudiannya diberi rawatan yang berpatutan mengikut diagnosis sepsis, iaitu rawatan piawai atau rawatan awal berasalkan sasaran untuk sepsis. Selepas 28 hari, pesakit akan dihubungi melalui telefon, emel atau laporan perubatan pesakit. Kemudian, pesakit akan dikatogorikan kepada golongan yang hidup atau mati. Dalam kajian ini, kepekatan laktat menujukan hubung kait yang erat dengan golongan yang mati berbanding dengan golongan yang hidup dalam ketiga-tigaan masa, iaitu jam 0, 1 dan 6 (p&lt;0.05). Akan tetapi, kadar pengurangan paras laktat tidak menunjukkan sebarang hubung kait dengan kadar kematian pesakit selepas 28 hari. Kesimpulannya, kepekatan laktat static merupakan petunjuk yang baik untuk meramalkan kadar kematian sepsis berbanding dengan kadar pengurangan paras laktat. Walaupun kadar pengurangan paras laktat tidak dapat membuktikan sebarang hubung kait dengan kadar kematian selepas 28 hari, kajian kami menunjukkan ia masih boleh dimanfaatkan sebagai pemantau yang baik semasa merawat pesakit sepsis.</p>\r\n', 1),
(1252, 1244, '<p>\r\n	Retinopati pramatang (ROP) adalah penyakit berkaitan dengan pertumbuhan saluran darah tidak matang pada bayi berat lahir rendah disebabkan oleh kekurangan oksigen pada retina yang sedang dalam pertumbuhan. Keadaan ini boleh membawa kepada risiko kebutaan. ROP selalunya terjadi pada kedua-dua mata, namun ada juga kes yang menunjukkan kelainan tahap keterukan penyakit di antara kedua mata. Kami melaporkan contoh kes yang mempunyai presentasi keterukan penyakit yang berbeza di antara kedua matanya dan rawatan ablasi retina hanya diberikan pada mata yang teruk sahaja. Kes ini memberi contoh salah satu kesan jangka panjang kepada pesakit yang mengalami ROP yang lebih teruk di dalam satu mata.</p>\r\n', 1),
(1255, 1247, '<p>\r\n	Laporan ini mengetengahkan peranan Perawatan komuniti asertif (ACT) dalam merawat sebuah keluarga yang kebanyakan ahlinya menghidap penyakit mental; skizofrenia. Kami melaporkan satu kes iaitu seorang wanita dalam usia pertengahan yang menghidapi penyakit skizofrenia resistan pada rawatan (TRS) yang tinggal bersama dua lagi ahli keluarganya yang menghidapi penyakit yang sama. Mereka dijaga oleh seorang penjaga yang menghidapi kemurungan major yang terkesan dari bebanan menanggung ahli keluarga yang sakit mental. ACT melalui pendekatannya yang holistik, terbukti dapat membantu mengurangkan risiko kemasukan ke hospital serta dapat membantu dalam penambahbaikan kawalan penyakit dan kualiti hidup keluarga ini.</p>\r\n', 1),
(1258, 1250, '<p>\r\n	Laporan ini mengetengahkan peranan Perawatan komuniti asertif (ACT) dalam merawat sebuah keluarga yang kebanyakan ahlinya menghidap penyakit mental; skizofrenia. Kami melaporkan satu kes iaitu seorang wanita dalam usia pertengahan yang menghidapi penyakit skizofrenia resistan pada rawatan (TRS) yang tinggal bersama dua lagi ahli keluarganya yang menghidapi penyakit yang sama. Mereka dijaga oleh seorang penjaga yang menghidapi kemurungan major yang terkesan dari bebanan menanggung ahli keluarga yang sakit mental. ACT melalui pendekatannya yang holistik, terbukti dapat membantu mengurangkan risiko kemasukan ke hospital serta dapat membantu dalam penambahbaikan kawalan penyakit dan kualiti hidup keluarga ini.</p>\r\n', 1),
(1264, 1256, '<p>\r\n	Diagnosa pembelahan aorta secara spontan jarang dilaporkan berlaku pada golongan dewasa muda, terutamanya tanpa aterosklerosis atau trauma. Lebih jarang lagi adalah kehadiran serentak pembelahan aorta dan aneurisma aorta dalam ketgori umur dewasa muda. Kami mengetengahkan suatu kes yang melibatkan pembelahan aorta dan aneurisma aorta yang dilihat berlaku pada seorang lelaki berusia 35 tahun. Computed tomographic angiography (CTA) menunjukkan pembelahan aorta jenis Stanford B dan pembelahan aorta daripada bifurkasi arteri iliac kanan sehingga arteri femoral. Aneurisma aorta juga dilihat melibatkan asal arteri subklavian. Prosedur hibrid melibatkan gabungan pembedahan terbuka dan endovaskular telah dijalankan.</p>\r\n', 1),
(1267, 1259, '<p>\r\n	Lavaj orogastrik telah dijalankan sejak 200 tahun yang lalu untuk pesakit yang telah diracun atau meracuni diri sendiri. Kekerapan procedur ini telah menurun sedekad yang lalu disebabkan factor risiko yang mengatasi manfaat. Seorang gadis remaja yang telah menelan sebotol racun perosak dalam tempoh masa yang sebelum pengosongan gastrik telah tiba di jabatan kecemasan kami. Pesakit itu diresusitasikan dan lavaj orogastrik telah dilakukan. Lima puluh cc bahan toksik telah disedut dan penawar toksin itu telah diberi. Pesakit kemudian telah dimasukkan ke Unit Rawatan Rapi dan seterusnya dibenarkan pulang dari hospital lima hari kemudian. Pengurusan saluran pernafasan yang betul, teknik yang betul, pemilihan yang betul bagi pesakit dan pemantauan yang mencukupi adalah penting bagi kejayaan lavaj orogastrik.</p>\r\n', 1),
(1269, 1261, NULL, NULL),
(1275, 1267, '<p>\r\n	-</p>\r\n', 1),
(1279, 1271, '<p>\r\n	Retinopati akibat dari radiasi (RR) ialah komplikasi yang sering berlaku kepada pesakit Nasopharyngeal Carcinoma (NPC). Kajian ini bertujuan untuk menentukan kaitan RR dengan dos radiasi yang diterima oleh pesakit NPC melalui pemeriksaan mata dan fundus fluorescein angiogram (FFA). Suatu kajian rentas lintang telah dilakukan melibatkan pesakit NPC yang telah menerima rawatan radiasi di Klinik Onkologi, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Sebanyak 82 mata dari 42 pesakit telah diperiksa dan prevalence RR didapati sebanyak 35.4%. Keterukan RR berkait rapat dengan dos radiasi kepada retina (Nilai Spearman correlation = 0.48, p&lt;0.001). Antara ciri- ciri RR yang dilihat melalui pemeriksaan FFA ialah salur darah telangiectatic (26.2%) dan capillary non-perfusion (14.3%). Retinal neovascularization dilihat berlaku kepada 10.7% dari mata yang diperiksa. Keterukan penglihatan didapati berkait rapat dengan keterukan RR dengan 26% daripada mata tersebut mempunyai penglihatan 6/18 atau lebih teruk. Lebih dari satu pertiga pesakit mendapat RR, dengan maculopathy radiasi sebagai penyebab yang paling kerap dilihat sebagai penyebab kehilangan penglihatan yang signifikan. FFA ialah alat yang sangat berguna untuk mengesan tanda-tanda awal RR dan maculpathy.</p>\r\n', 1),
(1290, 1282, NULL, NULL),
(1293, 1285, '<p>\r\n	Gegaran jantung merupakan hentakkan tumpul mengejut terhadap bahagian dada menyebab kematian. Walau bagaimanapun, terdapat sebahagian pesakit tidak memenuhi definisi tersebut. Kami melaporkan dua kes yang tidak memenuhi domain definisi tersebut. Dua mangsa lelaki mendapatkan rawatan di jabatan kecemasan dengan kesakitan dada sederhana selepas terlibat dalam kemalangan jalan raya. Mereka didapati paras ST yang meningkat pada led dada V2 dan V3 serta peningkatan paras kreatin kinase dan tropnin T. Elektrokardiogram tersebut balik ke paras biasa selepas 24 jam. Mereka discas dengan selamatnya selepas 24 jam rawatan dia wad sementara jabatan kecemasan. Definisi gegaran jantung (commotion cordis) konvensional dan lebam jantung agar tidak sesuai untuk mengambarkan keadaan kes-kes tersebut. Oleh itu, kita mencadangkan diagnosis gegaran jantuang setempat (fokus). Kita juga ingin menekankan kebolehan jabatan kecemasan untuk memantau dan merawat pesakit-pesakit golongan ini di wad sementara jabatan ini.</p>\r\n', 1),
(1299, 1291, '<p>\r\n	Kebelakangan ini, tanda-tanda kemurungan di kalangan remaja semakin meningkat. Kajian ini dijalankan untuk menentukan prevalens kemurungan dan faktor-faktor ramalan. Satu kajian keratan rentas telah dijalankan 191 remaja yang terpilih secara rawak dan terdiri daripada pelajar tingkatan empat dari lima buah sekolah menengah di negeri Selangor, Malaysia. Satu soal selidik yang terdiri daripada enam bahagian (A) demografi, (B) tahap kemurungan, (C) hubungan keluarga, (D) tahap sokongan rakan sebaya, (E) harga diri, dan (F) pencapaian akademik telah diedarkan. Prevalen kemurungan adalah 50.3%. Analisis regresi logistik mendapati, remaja yang mempunyai masalah dengan rakan-rakan adalah lebih cenderung untuk mengalami kemurungan berbanding dengan mereka yang tidak mempunyai masalah dengan rakan-rakan mereka (AOR 2.84, 95% CI 1.50, 5.36). Kajian selanjutnya perlu meneliti faktor-faktor lain seperti tekanan daripada guru-guru untuk mengukuhkan pemahaman kita mengenai kemurungan di kalangan remaja. Diharapkan hasil kajian ini berguna kepada pelbagai pihak yang mengambil berat tentang masalah ini.</p>\r\n', 1),
(1301, 1293, '<p>\r\n	Laporan kes ini menekankan tentang peranan kemurungan yang dimanifestasikan dalam bentuk homisid yang disusuli dengan percubaan bunuh diri (homicide-suicide). Kami melaporkan kes seorang pesakit yang dikatakan membunuh kekasihnya dan kemudian cuba untuk membunuh dirinya. Beliau pernah beberapa kali mengalami kegagalan dalam hubungan ditambah pula tekanan tempat kerja. Beliau diberi diagnosa kemurungan dan diberi ubat anti-kemurungan. Manifestasi homisid yang disusuli dengan percubaan bunuh diri akan dibincangkan daripada perspektif yang pelbagai.</p>\r\n', 1),
(1308, 1300, '<p>\r\n	Crab meat is a valuable nutrient source of proteins and functional lipids and is widely consumed. However the prevalence of crab allergy has increased over the past few years. In order to better understand crab allergy, it is necessary to identify crab allergens. The aim of this study was to compare the IgE-binding proteins of raw and cooked extracts of mud crab (Scylla serrata). Raw and cooked extracts of the mud crab were prepared. Protein profiles and IgE reactivity patterns were identified by sodium dodecyl sulfate polyacrylamide gel electrophoresis (SDS-PAGE) followed by immunoblotting using sera from 21 skin prick test (SPT) positive patients. In SDS-PAGE, 20 protein bands (12 to 250 kDa) were observed in the raw extract while the cooked extract demonstrated fewer bands. Protein bands between 40 to 250 kDa were sensitive to heat denaturation and no longer observed in the cooked extract. In immunoblotting experiments, raw and cooked extracts demonstrated 11 and 4 IgE-binding proteins, respectively, with molecular weights of between 23 and 250 kDa. A heat-resistant 36 kDa protein, corresponding to crab tropomyosin was identified as the major allergen of both extracts. In addition, a 41 kDa heat-sensitive protein believed to be arginine kinase was shown to be a major allergen of the raw extract. Other minor allergens were also observed at various molecular weights.</p>\r\n', 1),
(1309, 1293, 'Laporan kes ini menekankan tentang peranan kemurungan yang dimanifestasikan dalam bentuk homisid yang disusuli dengan percubaan bunuh diri (homicide-attempted suicide). Kami melaporkan kes seorang pesakit yang dikatakan membunuh kekasihnya dan kemudian cuba untuk membunuh dirinya. Beliau pernah beberapa kali mengalami kegagalan dalam hubungan ditambah pula tekanan tempat kerja. Beliau diberi diagnosa kemurungan dan diberi ubat anti-kemurungan. Kelangkaan kes homosid yang disusuli dengan percubaan bunuh diri boleh membantu pakar psikiatri untuk memahami fenomena ini sekali gus mencegahnya. \r\n', 1),
(1316, 1307, '<p>\r\n	Jerawat merupakan masaalah kulit yang dapat menjejaskan penampilan peribadi dan mengugat kualiti hidup seseorang. Satu kajian keratan rentas di kalangan pelajar-pelajar perubatan di Universiti Kebangsaan Malaysia telah dijalankan selama 12 bulan. Kajian ini bertujuan untuk mengenalpasti impak jerawat pada kualiti hidup, pengurusan diri dan cara-cara mengatasi masalah jerawat yang diamalkan oleh pelajar perubatan. Seramai 361 pelajar telah dipilih dengan menggunakan kaedah kluster berstrata persampelan rawak. Masalah jerawat telah digredkan dengan menggunakan Comprehensive Acne Severity Score (CASS), manakala impak jerawat pada kualiti hidup diukur dengan menggunakan Cardiff Acne Disability Index (CADI). Kaedah mengatasi masalah dan mendapatkan bantuan untuk masaalah jerawat diukur dengan menggunakan Acne Management Questionnaire. Purata markah CADI adalah 3.35 + 2.39, dengan impak yang rendah (83.8%, n=206) dan sederhana (14.6%, n=36) pada kualiti hidup. Pelajar perempuan dan pelajar dari kumpulan etnik bukan Melayu menunjukkan kadar impak kualiti hidup yang lebih tinggi (3.76 + 2.24, p=0.006 dan 3.79 + 2.59, p=0.018). Walaupun majoriti pelajar (87.4%, n=215) tahu bahawa jerawat boleh dirawat, mereka lebih gemar membincangkan masalah jerawat mereka dengan rakan-rakan (54.1%, n=133) daripada berunding dengan doktor (9.3%, n=23). Kajian ini menunjukkan bahawa pelajar memiliki jerawat gred yang rendah dan impak pada kualiti hidup adalah ringan. Walau bagaimanapun, usaha perlu ditingkatkan untuk menambahkan pengetahuan mereka mengenai pengurusan masalah jerawat dan membantu mengubah cara penelesaian masaalah kesihatan mereka. Langkah ini dapat membantu mengurangkan komplikasi akibat jerawat seperti parut, yang boleh menjejaskan kualiti hidup golongan bakal doktor ini.</p>\r\n', 1),
(1322, 1313, '<p>\r\n	Reseptor estrogen (ER), reseptor progesterone (PR) dan reseptor faktor ketumbuhan epidermal manusia 2 (HER2) immunohistokimia adalah penanda penting dan berperanan di dalam pengurusan pesakit yang menghidap neoplastik payudara. Di dalam kajian ini, kami membandingkan kadar konkordan ER, PR, dan HER2 diantara biopsi jarum teras dan biopsi eksais pada tisu neoplastik payudara melibatkan pesakit pesakit Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) yang di diagnosa diantara bulan Januari 2002 sehingga Disember 2012. Seramai 93 pesakit wanita yang menjalani biopsi jarum teras dan kemudiannya biopsi eksais termasuk di dalam kajian retrospektif ini. Kajian immunohistokimia telah digunakan untuk menentukan pewarnaan ER, PR dan HER2. Pewarnaan ER dan PR telah dianalisa menggunakan skor Allred (0 hingga 8) manakala analisa pewarnaan HER2 menggunakan sistem skor 0 hingga 3+. Skor keputusan dibanding antara kedua biopsy tersebut untuk menentukan kadar konkordan. Sebanyak 93 sampel berjaya dibandingkan diantara kedua biopsi tersebut untuk pewarnaan immunohistokimia ER dan PR. Kadar konkordan ER adalah sebanyak 80 kes (86%) dan 13 kes (14%) bagi yang tidak konkordan. Kadar konkordan PR adalah sebanyak 82 kes (88.2%) dan 11 kes (11.8%) bagi yang tidak konkordan. Sebanyak 87 sampel telah dibandingkan untuk kajian pewarnaan immunohistokima HER2 dan kadar konkordan adalah sebanyak 62 kes (71.3%) dan 25 kes (28.7%) bagi yang tidak konkordan. Di dalam kajian ini, kadar konkordan diantara biopsi tisu jarum teras dan biopsi eksais bagi ER dan PR adalah tinggi. Manakala, kadar konkordan untuk pewarnaan HER2 adalah kurang konsisten. &nbsp;Kesimpulannya, analisa immunohistokimia untuk biopsi tisu jarum teras dapat meramal keputusan penanda pada tisu biopsy eksais di dalam penyakit neoplastik payudara.</p>\r\n', 1),
(1326, 1317, '<div>\r\n	Objektif kajian ialah untuk menentukan kesan program latihan papan Wobble ke atas daya keseimbangan para atlet yang mengalami ketakstabilan fungsi pergelangan kaki (FAI). Sebanyak 30 subjek (n=30) yang mengalami FAI direkrut berdasarkan kriteria rangkuman dan eksklusi. Kesemua subjek dinilai daya keseimbangan menggunakan ujian &nbsp;dirian anggota tunggal (single limb stance) dan ujian jangkauan fungsi dan didedahkan kepada program latihan papan Wobble sebanyak lima sesi seminggu selama empat minggu. Ujian t berpasangan digunakan untuk membandingkan min data pra dan post-ujian dan aras signifikan ditetapkan pada nilai 0.05. Pada ujian dirian anggota tunggal, para atlet mendapat min skor post-latihan yang tinggi [26.4] berbanding pra-latihan. Perbezaan disahkan dengan nilai &lsquo;t&rsquo; yang diperolehi [16.00]. Bagi ujian jangkauan fungsi, para atlet memperolehi min skor post-latihan yang tinggi [15.88] berbanding pra-latihan [1.26]. Perbezaan disahkan dengan nilai &lsquo;t&rsquo; yang diperolehi [12.54]. Ini menunjukkan bahawa latihan papan Wobble meningkatkan keseimbangan dinamik dan statik para atlet yang mengalami FAI. Secara kesimpulannya, latihan papan Wobble mungkin efektif dalam meningkatkan keseimbangan para atlet yang mengalami FAI.</div>\r\n', 1),
(1329, 1320, '<div>\r\n	Retinopati akibat dari radiasi (RR) ialah komplikasi yang sering berlaku kepada pesakit Nasopharyngeal Carcinoma (NPC). Kajian ini bertujuan untuk menentukan kaitan RR dengan dos radiasi yang diterima oleh pesakit NPC melalui pemeriksaan mata dan fundus fluorescein angiogram (FFA). &nbsp;Suatu kajian rentas lintang telah dilakukan melibatkan pesakit NPC yang telah menerima rawatan radiasi di Klinik Onkologi, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Sebanyak 82 mata dari 42 pesakit telah diperiksa dan prevalence RR didapati sebanyak 35.4%. Keterukan RR berkait rapat dengan dos radiasi kepada retina (Nilai Spearman correlation = 0.48, p&lt;0.001). Antara ciri- ciri RR yang dilihat melalui pemeriksaan FFA ialah salur darah telangiectatic (26.2%) dan capillary non-perfusion (14.3%). Retinal neovascularization dilihat berlaku kepada 10.7% dari mata yang diperiksa. Keterukan penglihatan didapati berkait rapat dengan keterukan RR dengan 26% daripada mata tersebut mempunyai penglihatan 6/18 atau lebih teruk. Lebih dari satu pertiga pesakit mendapat RR, dengan maculopathy radiasi sebagai penyebab yang paling kerap dilihat sebagai penyebab kehilangan penglihatan yang signifikan. FFA ialah alat yang sangat berguna untuk mengesan tanda-tanda awal RR dan maculpathy.</div>\r\n', 1),
(1332, 1323, '<div>\r\n	Di peringkat global, strok adalah punca utama penyakit kecacatan jangka panjang. Ketidakupayaan di kalangan pesakit strok mempengaruhi aktiviti kehidupan harian mereka. Tujuan terapi pemulihan adalah untuk membantu mangsa-mangsa strok untuk mendapatkan kembali keupayaan berfungsi. Kajian ini bertujuan untuk menentukan hubungan antara tempoh masa strok dengan status keupayaan berfungsi &nbsp;dalam kalangan pesakit strok di salah sebuah &nbsp;hospital pengajaran di Kuala Lumpur. Satu kajian keratan rentas dijalankan, melibatkan 109 orang pesakit strok yang menghadiri Klinik Pemulihan dalam tempoh masa empat bulan. Data mengenai tempoh masa &nbsp;strok diambil daripada rekod &nbsp;pesakit manakala keupayaan berfungsi dinilai melalui Indeks Barthel Diubahsuai (MBI). Keputusan menunjukkan min umur pesakit &nbsp;adalah 61 tahun (SD = 13,86, julat: 22-87 tahun), dengan 118 lelaki dan 81 pesakit wanita yang mempunyai tempoh masa dengan &nbsp;median 12 bulan selepas strok &nbsp;(julat: 1-79 bulan). Prevalens 123 (63.1%) daripada mangsa strok didapati tidak bergantung dalam aktiviti kehidupan harian mereka. Ujian Chi-kuasa dua menunjukkan terdapat hubungan yang signifikan di antara tempoh masa strok dengan tahap kebergantungan berfungsi, &chi;2(2, n = 195) = 6,455, p &lt;0.05, phi = 0,182. Pesakit dalam tempoh masa strok daripada 13-24 bulan adalah lebih berdikari (52.1) daripada &le;12 bulan dan &ge; 24 bulan.</div>\r\n', 1),
(1342, 1333, '<div>\r\n	Mercu tanda anatomi foramen nutrien penting dalam prosedur pembinaan semula kaki yang melibatkan penggunaan tulang metatarsal. Objektif kajian ialah untuk mengkaji morfologi foramina nutrien di tulang metatarsal dan mendapatkan indeks foramina. Kajian ini melibatkan 271 metatarsal manusia yang bukan berpasangan. Umur dan jantina metatarsal tidak diketahui. Foramina nutrien setiap tulang diperiksa untuk menentukan topografi dan nombor. Pengindeksan foramina dilakukan menggunakan formula Hughes. Morphometri kajian ini telah dijalankan dengan penggunaan &lsquo;Vernier caliper&rsquo; digital. Hasil pemerhatian mendapati 79.3% daripada metatarsal mempunyai foramen nutrien tunggal, 9.2% mempunyai dua foramen, 1.1% mempunyai tiga foramen dan 10.4% metatarsal tidak ada foramen. Min indeks foramina daripada metatarsal pertama, metatarsal kedua, metatarsal ketiga, metatarsal keempat dan metatarsal kelima ialah 65.3, 43.6, 44.4, 45.3 dan 47.5 masing-masing mengikut turutan. Dapat diperhatikan bahawa kebanyakan metatarsal mempunyai foramen nutrien tunggal. Lokasi foramen nutrien berubah dari metatarsal 1 hingga 5. &nbsp;Foramina metatarsal 1 dan metatarsal 5 diperhatikan di sebelah sisi mereka dan pada metatarsal yang lain; ia lazim diperhatikan di permukaan medial. Pengagihan morfologi tentang foramina nutrien adalah amat diperlukan dalam sesetengah pembedahan seperti penggunaan tulang metatarsal dalam pembinaan semula kaki. Data morfologi juga amat penting dalam pembedahan plastik seperti kes operasi mikrovaskular tisu tulang.</div>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(1344, 1335, '<div>\r\n	Kebelakangan ini, tanda-tanda kemurungan dalam kalangan remaja semakin meningkat. Kajian ini dijalankan untuk menentukan prevalens kemurungan dan faktor-faktor ramalan. Satu kajian keratan rentas telah dijalankan 191 remaja yang terpilih secara rawak dan terdiri daripada pelajar tingkatan empat dari lima buah sekolah menengah di negeri Selangor, Malaysia. Satu soal selidik yang terdiri daripada enam bahagian (A) demografi, (B) tahap kemurungan, (C) hubungan keluarga, (D) tahap sokongan rakan sebaya, (E) harga diri, dan (F) pencapaian akademik telah diedarkan. Prevalen kemurungan adalah 50.3%. Analisis regresi logistik mendapati, remaja yang mempunyai masalah dengan rakan-rakan adalah lebih cenderung untuk mengalami kemurungan berbanding dengan mereka yang tidak mempunyai masalah dengan rakan-rakan mereka (aOR 2.84, 95% CI 1.50, 5.36). Kajian selanjutnya perlu meneliti faktor-faktor lain seperti tekanan daripada guru-guru untuk mengukuhkan pemahaman kita mengenai kemurungan di kalangan remaja. Diharapkan hasil kajian ini berguna kepada pelbagai pihak yang mengambil berat tentang masalah ini.</div>\r\n', 1),
(1349, 1340, '<p>\r\n	epercayaan pesakit terhadap pengurusan kesakitan boleh menjejaskan hasil pengurusan kesakitan dan kualiti hidup. Tujuan kajian ini dijalankan adalah untuk menentukan kesan pakej pendidikan pra-operasi sakit ke arah kepercayaan kesakitan dalam kalangan pesakit-pesakit yang menjalani pembedahan ortopedik di hospital tertiari. Reka bentuk kajian &lsquo;pre-test pos-test&rsquo; satu-kumpulan &nbsp;dijalankan pada pesakit-pesakit untuk menentukan impak pakej pendidikan tentang kesakitan terhadap skor &lsquo;post-test&rsquo; kepercayaan tentang kesakitan dalam kalangan pesakit &nbsp;yang menjalani pembedahan ortopedik. Tiga puluh responden telah diambil menggunakan &lsquo;convenience sampling&rsquo;. Pendidikan tentang kesakitan sebelum pembedahan berkaitan dengan pengurusan kesakitan diberikan kepada respons.Skor kepercayaan pra-operasi dan pos-operasi sakit, pengurusan markah dan kesan-kesan sampingan telah diukur dengan menggunakan borang soal-selidik halangan (BQ-13). Keputusan melaporkan perbezaan yang signifikan antara skor &lsquo;pre-test&rsquo; (Mean = 41.87, Standard Deviation = 11.467) dan &lsquo;post-test&rsquo; (Mean = 34,80, Standard Deviation = 13.026) kepercayaan terhadap kesakitan (t = 2.84, p =0.004). Terdapat juga perbezaan yang signifikan antara skor &lsquo;pre-test&rsquo; (Mean = 37.10, Standard Deviation = 10.610) dan skor &lsquo;post-test&rsquo; (Mean = 30.80, Standard Deviation = 11.424), pengurusan kesakitan (t = 3.856, p =0.0005). Responden dan jantina (t =-2.403, p =0.023) dan bangsa (F = 5.038, p =0.014) melaporkan perbezaan yang signifikan iaitu p &lt; 0.05. Bagaimanapun, tidak terdapat perbezaan yang signifikan antara tahap pendidikan, etnik, sejarah pembedahan terlebih dahulu dengan kepercayaan kesakitan dengan p &gt; 0.05. Kesimpulannya, terdapat &nbsp;kesan positif daripada pakej pendidikan kesihatan berkenaan kepercayaan kesakitan dan pengurusan kesakitan responden yang telah menjalani pembedahan ortopedik di hospital tertiari. Pengukuhan program pendidikan kesakitan adalah penting untuk mencapai matlamat pengurusan kesakitan pos-operasi yang optimum.</p>\r\n', 1),
(1352, 1343, '<div>\r\n	Sakit adalah sensasi yang tidak menyenangkan dan boleh menyebabkan masalah fizikal dan psikologi untuk pesakit. Walaupun intervensi secara farmakologi digunakan untuk mengurangkan kesakitan, ia masih kekal sebagai salah satu isu selepas pembedahan. Penggunaan terapi muzik sebagai intervensi bukan farmakologi boleh memberikan kesan terhadap kesakitan selepas pembedahan dan keperluan sakit untuk mendapatkan analgesik. Tujuan kajian ini adalah untuk menentukan kesan terapi muzik terhadap kesakitan selepas Pembedahan Elektif Penggantian Lutut secara total. Kajian ini membandingkan penggunaan analagesik selepas pembedahan oleh pesakit dalam dua kumpulan. Kajian ini juga mendedahkan kesan terapi muzik terhadap kumpulan etnik yang berbeza. Kajian berbentuk kuasi-experimental dengan persempalan berkemudahan mengambil pesakit berumur 49-76 yang telah menjalani pembedahan penggantian lutut secara keseluruhan di PPUKM dari Mei sehingga Disember 2012. Sebanyak 40 pesakit dipilih secara rawak dalam salah satu daripada dua kumpulan dengan mengunakan teknik sampul surat tertutup. Kumpulan eksperimen telah mendengar muzik selama lima hari selapas pembedahan dan diberi analgesik serta kumpulan kawalan hanya menggunakan intervensi farmakologi sahaja. Skor kesakitan telah diukur dengan mengunakan soal selidik kesakitan McGill bentuk ringkas pada waktu rehat di katil pada hari pertama, ketiga dan kelima selepas pembedahan. Analisis (Mann-Whitney) menunjukkan kumpulan eksperimen mengalami kesakitan yang berkurangan pada hari pertama dan kelima berbanding kumpulan kawalan pada tahap 0.05 menggunakan PRI, VAS dan PPI. Analisis (Friedman) menunjukkan sampel dalam kumpulan mengalami penurunan kesakitan ketara dari masa ke masa pada tahap 0.05 menggunakan PRI, VAS dan PPI. Analisis (Mann-Whitney) menunjukkan tidak terdapat perbezaan yang signifikan dalam menggunakan analgesik dalam milligram antara dua kumpulan ini dalam tempoh lima hari selepas pembedahan namun min menunjukkan kumpulan eksperimen menggunakan kurang analgesik berbanding kumpulan kawalan. Analisis (Krusl-Wallis) menunjukkan tidak terdapat perbezaan yang ketara dalam kesan terapi muzik selepas pembedahan antara kaum (Melayu, Cina dan India) menggunakan VAS. Terapi muzik merupakan intervensi yang berkesan ringkas mudah, dipadah, selamat, murah dan efektif untuk pengurusan kesakitan selepas pembedahan. Gabungan intervensi bukan farmakologi dan analgesik boleh mengurangkan kesakitan lebih daripada hanya menggunakan intervensi farmakologi sahaja. Pengurusan kesakitan adalah satu peranan penting dalam kejururawatan dan jururawat boleh menggunakan terapi muzik sebagai intervensi secara langsung dalam mengurangkan kesakitan pesakit.</div>\r\n', 1),
(1355, 1346, '<p>\r\n	Gegaran jantung merupakan hentakan tumpul, mengejut terhadap bahagian dada yang boleh menyebab kematian. Walaubagaimanapun, terdapat sebahagian pesakit tidak memenuhi definisi tersebut. Kami melaporkan dua kes yang tidak memenuhi domain definisi tersebut. Dua mangsa lelaki mendapatkan rawatan di Jabatan Kecemasan dengan kesakitan dada sederhana selepas terlibat dalam kemalangan jalan raya. Didapati paras ST meningkat pada led dada V2 dan V3 serta peningkatan paras kreatin kinase dantropnin T. &nbsp;Elektrokardiogram tersebut balik ke paras biasa selepas 24 jam. Mereka discas dengan selamatnya selepas 24 jam rawatan di wad sementara Jabatan Kecemasan. Definisi gegaran jantung (commotion cordis) konvensional dan lebam jantung adalah tidak sesuai untuk mengambarkan keadaan kes-kes tersebut. Oleh itu, kami mencadangkan diagnosis gegaran jantuang setempat (fokus). Kami juga ingin menekankan kebolehan Jabatan Kecemasan untuk memantau dan merawat pesakit-pesakit golongan ini di wad sementara jabatan ini. &nbsp;</p>\r\n', 1),
(1358, 1349, NULL, NULL),
(1364, 1355, 'Jangkitan kulat dalam mata boleh mengancam penglihatan seseorang individu, terutamanya seandainya kulat berkenaan turut berada di dalam aliran darah. Komplikasi tersebut boleh dicegah jika ia dikenalpasti awal dan diberi rawatan secepat mungkin. Kami mengetengahkan satu kes yang telah berlaku pada seorang wanita kencing manis berusia 64 tahun yang kerapkali masuk ke wad disebabkan jangkitan berulang dalam saluran kencing. Jangkitan kulat telah ditemui dalam darah pada peringkat awal kemasukan namun tidak dirawat. Pemeriksaan mata ketika kemasukan wad selanjutnya mendapati kulat berkenaan turut berada di dalam mata. Maka, gabungan rawatan terhadap kulat melalui darah dan melalui selai mata diberikan dan penglihatan beliau pulih semasa beliau dibenarkan keluar wad.', 1),
(1716, 1707, 'Penggunaan mesin ultrasound telah menjadi lebih meluas dengan peredaran masa di jabatan kecemasan. Mesin ultrasound di gunakan untuk mengenalpasti cecair di bahagian abdomen, cecair di dalam lapisan luar jantung, angin di dalam rongga dada, dan keadaan hidrasi pesakit. Penyelidikan ini bertujuan menilai kebolehan alat ultrasound untuk mengenalpasti patah tulang berbanding dengan mesin x ray. Penyelidikan ini adalah di satu pusat dengan kaedah ‘keratan rentas – cross sectional). Dalam jangkamasa bulan ogos 2014 hingga November 2014, sejumlah 46 pesakit menyertai penyelidikan ini, menghasilkan 75 imej. Bagi pesakit yang memberi kebenaran, siasatan ultrasound dilakukan, dan kemudian dibanding dengan laporan x ray mengenai kewujudan patah tulang. Analisa SPSS digunakan untuk menentukan tahap kepekaan (sensitivity) dan pengkhususan (specificity) alat ultrasound berbanding mesin x ray. Kepekaan ultrasound adalah 72%(95% CI, 50.6% - 87.9%)  dan pengkhususan ultrasound adalah 80% (95%CI: 66.3 – 90%) berbanding alat x ray untuk mengenalpasti patah tulang. Analisa kappa menunjukan bahawa persetujuan antara pemerhati adalah sederhana (0.5). Penyelidikan ini menunjukkan bahawa alat ultrasound boleh diguna untuk menghantar pulang pesakit dari kawasan triaj jabatan kecemasan.', 1),
(6045, 6036, '<p>\r\n	-</p>\r\n', 1),
(6052, 6043, '<p>\r\n	Ketam adalah sumber nutrien berharga iaitu protein dan lipid berfungsi yang diambil secara meluas. Walau bagaimanapun, prevalen alergi terhadap ketam telah meningkat beberapa tahun kebelakangan ini. Dalam usaha untuk lebih memahami alergi terhadap ketam, adalah penting untuk mengenal pasti alergen-alergen ketam. Tujuan kajian ini adalah untuk membandingkan protein pengikat-IgE pada ekstrak ketam lumpur (Scylla serrata) mentah dan yang dimasak. Ekstrak ketam lumpur mentah dah yang dimasak telah disediakan. Profil protein dan corak kereaktifan-IgE telah dikenalpasti dengan menggunakan elektroforesis gel natrium dodesil sulfat berpoliakrilamida (SDS-PAGE), diikuti dengan pemblotan imuno menggunakan serum daripada 21 orang pesakit yang positif dalam ujian cucuk kulit (SPT). Dalam SDS-PAGE, sebanyak 20 jalur protein (12 hingga 250 kDa) telah diperhatikan dalam ekstrak ketam mentah manakala ekstrak yang dimasak menunjukkan sedikit jalur. Jalur protein antara 40 hingga 250 kDa adalah sensitif terhadap penyahaslian haba dan tidak dapat diperhatikan lagi dalam ekstrak yang dimasak. Dalam eksperimen pemblotan imuno, ekstrak mentah dan yang dimasak masing-masing menunjukkan 11 dan 4 protein pengikat-IgE, dengan berat molekul antara 23 dan 250 kDa. Protein 36 kDa yang tahan haba, yang sepadan dengan tropomiosin ketam telah dikenal pasti sebagai alergen major pada kedua-dua ekstrak. Sebagai tambahan, protein 41 kDa yang sensitif haba yang dipercayai adalah arginine kinase telah dikenal pasti sebagai alergen major dalam ekstrak mentah. Alergen minor yang lain juga telah diperhatikan pada pelbagai berat molekul.</p>\r\n', 1),
(6035, 6026, 'Objektif kajian ini adalah untuk mengenalpasti faktor-faktor yang berkaitan dengan kehadiran semula pada peringkat awal pesakit asma yang didiscajkan daripada Jabatan Kecemasan. Ia adalah satu kajian kohort retrospektif untuk pesakit-pesakit yang berumur 12 tahun atau lebih dalam tempoh 1 bulan (1 Mac 2014 - 31 Mac 2014) yang telah dirawat untuk asma akut dan didiscajkan daripada ED Hospital Umum Sarawak. Seramai 397 pesakit memenuhi kriteria sampel  dan daripada jumlah ini, 13.9% telah hadir semula dalam tempoh 2 minggu. Daripada kes-kes kehadiran semula , 9.1 % dimasukkan ke wad. Kadar perskripsi kortikosteroid oral adalahi rendah ( 24.9 %) dan melarikan diri daripada hospital adalah tinggi (25.1 %). Pesakit yang telah melarikan diri dari jabatan kecemasan dan jangkitan mereka berkadar terus dengan ketibaan semula ke jabatan kecemasan. ', 1),
(6042, 6033, NULL, NULL),
(6053, 6044, '<p>\r\n	Strok sering dikaitkan dengan ketidakupayaan jangka panjang. Ia memberi kesan negatif kepada pesakit strok akibat gangguan fizikal dan neuropsikologi. Ketidakupayaan dalam kalangan pesakit strok akan mendorong mereka mengalami kemurungan. Kajian ini adalah bertujuan untuk menentukan hubungan antara ciri-ciri klinikal dan tahap kemurungan dalam kalangan pesakit strok di Hospital Pengajaran di Kuala Lumpur. Kajian keratan rentas ini melibatkan 195 pesakit strok yang menghadiri rawatan susulan di Klinik Pemulihan Hospital Pengajaran bagi tempoh 4 bulan. Maklumat yang dikumpul termasuk sosio-demografi, data klinikal (tempoh pos strok, jenis strok dan keterukan strok) dan tahap kemurungan. Beck Depression Inventory (BDI) digunakan untuk menilai tahap kemurungan pesakit strok. Keputusan menunjukkan min umur pesakit adalah 61 tahun (SD = 13,86, julat: 22-87 tahun), dengan 118 dan 81 pesakit wanita yang mempunyai tempoh masa dengan median 12 bulan selepas strok (julat: 1-79 bulan). Kajian ini menunjukkan 116 (59.5 %) mengalami kemurugan. Ujian Chi-kuasa dua untuk tahap keterukan strok nyata berkaitan dengan kemurungan, &chi;2 (2, n = 195) = 28,724, p &lt;0.001, phi = 0,384. Kajian ini menunjukkan pesakit yang mengalami jenis strok ringan mempunyai kemurungan kurang (44.0%) berbanding dengan strok sederhana (81.7%) dan strok yang teruk (87.5%)</p>\r\n', 1),
(6057, 6048, '<p>\r\n	Penggunaan mesin ultrasound telah menjadi lebih meluas dengan peredaran masa di Jabatan Kecemasan. Penyelidikan ini bertujuan menilai kebolehan alat ultrasound untuk mengenalpasti kepatahan tulang berbanding dengan mesin X-ray di dalam situasi berbeza seperti di kawasan triaj. Penyelidikan ini adalah di satu pusat dengan kaedah keratan rentas. Dalam tempoh Ogos 2014 hingga November 2014, sejumlah 46 pesakit menyertai penyelidikan ini menghasilkan 75 imej. Bagi pesakit yang memberi kebenaran, siasatan ultrasound dilakukan, dan kemudian dibanding dengan laporan X-ray mengenai kewujudan patah tulang. Analisa SPSS digunakan untuk menentukan tahap kepekaan (sensitivity) dan pengkhususan (specificity) alat ultrasound berbanding mesin X-ray. Kepekaan ultrasound adalah 72%(95% CI, 50.6% - 87.9%) dan pengkhususan ultrasound adalah 80% (95%CI: 66.3 &ndash; 90%) berbanding alat X-ray untuk mengenalpasti patah tulang. Analisa kappa menunjukan bahawa persetujuan antara pemerhati adalah sederhana (0.5). Penyelidikan ini menunjukkan bahawa penggunaan alat ultrasound sebagai alat triaj untuk menghantar pulang pesakit dari kawasan triaj Jabatan Kecemasan tidak sensitif dan perlu kepada penilaian dan pertimbangan selanjutnya.</p>\r\n', 1),
(6059, 6050, '<div>\r\n	Pendarahan sel darah bayi jenis rhesus (Rh) positif ke dalam aliran darah ibu berdarah Rh negatif boleh berlaku semasa mengandung dan semasa proses kelahiran.Perbezaan jenis darah ini menyebabkan ibu menghasilkan Rh antibody dan menyebabkan pemusnahan sel pramatang pada darah bayi. Ujian Kleihauer adalah kaedah biasa digunakan untuk mengukur pendarah sel darah bayi. Walaubagaimanapun tiada penyelarasan khusus antara makmal bagi ujian ini dan keputusannya kurang tepat. Teknik sitometri aliran adalah kaedah baru menggunakan anti HbF monoclonal. Ia boleh mengkaji lebih banyak sel, lebih sensitif dan tepat. Tujuan kajian ini adalah untuk mengukur pendarahan sel darah bayi di dalam aliran darah ibu menggunakan dua teknik tersebut dan menentukan tahap korelasi antara keduanya. 98 sampel darah ibu mengandung lebih dari 20 minggu dan selepas bersalin telah dianalisa. Kajian mendapati tahap korelasi yang sederhana antara dua kaedah ujian, r = 0.655 (p&lt;0.05) dan tahap keharmonian adalah 66.3%. Walaubagaimanapun, kaedah sitometri aliran member bacaan yang lebih tinggi untuk 70.4% kes berbanding kaedah Kleihauer. Pemerhatian ini mungkin disebabkan oleh cahaya otoflores yang dihasilkan oleh kontaminasi sel darah putih atau penggunaan anti HbF yang kurang spesifik berbanding anti-D monoclonal. Ia boleh juga berpunca dari jenis fluorokrom yang digunakan. Oleh itu, untuk meningkatkan ketepatan keputusan ujian, analisan dua label glycophorin A yang spesifik untuk sel darah merah dan perbandingan antara anti HbF dan anti-D adalah disyorkan.</div>\r\n', 1),
(6061, 6052, '<div>\r\n	Papillary Nasopharyngeal Adenocarcinoma Gred Rendah (LGPAC) adalah sejenis tumor berasal dari mukosa yang jarang berlaku berbanding kanser nasofarinks jenis &ldquo;keratinized&rdquo;. Secara histology, terdapat diferensiasi jenis papillari yang juga boleh didapati dalam kes metastatik karsinoma papilari tiroid. Keadaan ini menyebabkan cabaran bagi pihak patologi untuk membezakan antara kes primari atau metastatik. Thyroid Transcription Factor-1 (TTF-1) diekspreskan dalam subset Papillary Nasopharyngeal Adenocarcinoma dan ini merupakan alat diagnostic yang berguna. LGPAC adalah tumour malignan bersifat benign. Diagnosis awal beserta pembedahan membuang keseluruhan tumour melalui cara konvensional atau endoskopik boleh memberikan prognosis bagus dan risiko berlakunya lagi tumour ini adalah rendah.&nbsp;</div>\r\n', 1),
(6062, 6053, '<p>\r\n	Laporan ini menekankan tentang peranan kemurungan yang dimanifestasikan dalam bentuk homisid yang disusuli dengan percubaan bunuh diri (homicide-attempted suicide). Kami melaporkan kes seorang pesakit yang dikatakan membunuh kekasihnya dan kemudian cuba untuk membunuh dirinya. Beliau pernah beberapa kali mengalami kegagalan dalam hubungan ditambah pula tekanan tempat kerja. Beliau didiagnos mengalami kemurungan dan diberi ubat anti-kemurungan. Kelangkaan kes homosid yang disusuli dengan percubaan bunuh diri boleh membantu pakar psikiatri untuk memahami fenomena ini sekaligus mencegahnya.</p>\r\n', 1),
(6066, 6057, '<div>\r\n	Laporan kes &nbsp;ini mengetengahkan Syndrom &lsquo;Charles Bonnet&rsquo; sebagai faktor dorongan &nbsp;pembentukkan penyakit neurokognitif utama disebabkan oleh penyakit Alzheimers di kalangan warga tua yang juga mempunyai masalah penglihatan dan juga diagnosis penyakit yang lain. Kami melaporkan kes tentang seorang wanita tua yang tiada masalah kesihatan dan perubatan buat pertama kalinya hadir ke klinik psikiatri dengan halusinasi visual kompleks di mana pesakit melihat imej manusia yang jelas dan objek selama 18 bulan. Selepas lebih kurang setahun, terdapat kemerosotan kognitif secara beransur-ansur dengan gejala psikotik seperti halusinasi suara dan delusi persekutori. Tiada gejala afektif atau obsessi dilaporkan. Beliau mempunyai kurang kesedaran terhadap penyakit beliau. Pemeriksaan mata menunjukkan kemerosatan akuiti visual di kedua-dua belah mata. Selain daripada itu, pemeriksaan fizikal adalah didapati normal. Beliau menerima rawatan ubat-ubatan Rivastigmine patch 4.6 mg/24 jam and Zydis 10 mg waktu malam. Kemerosotan kognitif dan gejala psikotik secara beransur-ansur pulih dalam tempoh masa 2 minggu sejak menerima rawatan dan juga rawatan susulan di klinik pesakit luar. Beliau juga semakin menyedari akan penyakit beliau. Syndrom &lsquo;Charles Bonnet&rsquo; boleh menjadi faktor pencetus dan pendorong masalah penyakit neurokognitif disebabkan oleh penyakit &lsquo;Alzheimer&rsquo;s&rsquo; di kalangan warga tua yang mempunyai masalah penglihatan yang sering terlepas pandang, &nbsp;atau tersilap diagnosia dan justeru itu kurang dilaporkan.</div>\r\n', 1),
(6073, 6064, '<p>\r\n	Kajian genetik terdahulu menunjukkan bahawa kelangsungan hidup dan proliferasi parasit Plasmodium falciparum bergantung kepada penggunaan purin utama dalam hos. Plasmodium falciparum, agen penyebab kepada jenis malaria paling merbahaya pada manusia, tidak mempunyai enzim yang diperlukan untuk sintesis purin secara de novo. Berdasarkan kepada sasaran kemoterapeutik yang novel, analisa hipotetikal protein pengangkut nukleosida/nukleobas, hasil dari gen PfNT3 (PF14_0662) dalam parasit P. falciparum, dilakukan secara lokalisasi. Melalui immunobloting, immunoflorescen dan lokalisasi mikroskopik immunoelektron, lokasi PfNT3 didemonstrasi dengan menggunakan anti-serum poliklonal pada kultur in-vitro parasit P. falciparum yang dibiakkan dalam sel darah merah manusia. Protin PfNT3, hasil translasi gen PfNT3, dikesan pada peringkat cincin intraeritrosit, tropozit dan skizon. Tumpuan utama PfNT3 adalah pada membran plasma parasit (MPP). Pengangkut putatif nukleosid endogenous PfNT3 dengan lokasi utama di MPP boleh bertindak bukan sahaja sebagai tapak jalan untuk sasaran analog purin/ agen sitotoksik bagi intraselular parasit , tetapi juga bertindak sebagai sasaran dadah. Sebagai genom terkod, protin pengangkut vital boleh dihalang daripada diekspresi melalui gen &lsquo;silencing&rsquo; dan ini mengesahkanya sebagai sasaran dadah yang novel.</p>\r\n', 1),
(6078, 6069, '<p>\r\n	Trombus di dalam salur vena kava inferior biasanya memberi gambaran kanser hati tahap terakhir. Sebelum ini, tidak ada sebarang rawatan yang dapat memberikan keputusan yang memberangsangkan. Tetapi dengan kaedah rawatan endovaskular, harapan baru mungkin dapat dikecapi. Kami ingin mempertengahkan satu kes dimana terapi endovaskular menggunakan AngioJet&reg; rheolytic system (Possis Medical, Minneapolis, MN, USA), dapat menunjukkan keputusan yang positif.</p>\r\n', 1),
(6081, 6072, '<p>\r\n	Kesakitan pada bahangian bawah dan kanan perut atau abdomen sentiasa disalah diagnosa walaupun doktor bedah kini bergantung pada teknik pengimejan yang terbaru. Ini adalah lebih mencabar terutamanya pada pesakit wanita dimana kesilapan diagnosa kesakitan pada bahagian bawah dan kanan abdomen adalah sebanyak 40%. Seorang wanita berumur 66 tahun hadir dengan sakit pada bahagian bawah dan kanan abdomen selama seminggu. Pada mulanya, semasa pemeriksaan dijalankan didapati terdapat ketumbuhan di bahagian perut yang disyaki barah. Skan Computed Tomography menunjukkan kasakitan abdomen kemungkinan berpunca daripada koleksi nanah. Namun demikian, prosedur jarum aspirasi menghasilkan bahan berunsur najis berwarna coklat yang mencadangkan penyakit diverticulitis. Pembedahan penerokaan perut dan membuang appendik mendedahkan appendik yang bernanah dan bukan barah. Kesimpulannya, apabila penilaian klinikal dan pengimejan tidak dapat disimpulkan, penerokaan laparotomi abdomen adalah wajar terutamanya jika barah disyaki.</p>\r\n', 1),
(6087, 6078, '<p>\r\n	Fistula gaster-peritoneum merupakan salah satu komplikasi pembedahan gastrektomi lengan laparoskopik yang jarang berlaku tetapi memberi kesan morbidity dan mortaliti yang signifikan. Kes ini melibatkan seorang pesakit lelaki berumur 42 tahun yang mempunyai masalah obesiti morbid dan telah menjalani pembedahan gastrektomi lengan laporoskopik. Setelah pembedahan, beliau mengalami masalah gastric kronik dan refluks berpanjangan. Siri gaster usus menunjukkan terdapat fistula yang berhubung di antara gaster dengan hemi-diafragma kiri dan kawasan sekeliling limpa.</p>\r\n', 1),
(6092, 6083, NULL, NULL),
(6093, 6084, NULL, NULL),
(6106, 6097, NULL, NULL),
(6111, 6102, NULL, NULL),
(6309, 6300, NULL, NULL),
(6556, 6547, '<p>\r\n	Penggunaan teknologi sebagai salah satu kaedah pengajaran terkenal dalam bidang perubatan . Salah satu kaedah yang digunakan ialah pembelajaran dengan video. Pengunaan video sebagai bahan pengajaran bagi skil kecemasan telah dilaksanakan. Walau bagaimanapun, kajian ke atas persepsi menggunakan kaedah ini ke arah pembelajaran skil kecemasan masih belum dikaji. Fokus kajian ini adalah pendekatan kualitatif dengan tumpuan kepada empat kumpulan focus setelah mereka megikuti sesi pembelajaran skil kecemasan dengan video. Seramai 20 orang peserta dibahagikan kepada empat kumpulan. Mereka terlibat dalam perbincangan setelah mengikuti penmbelajaran melalui video. Hasil kajian ini menunujukan bahawa terdapat tiga tema utama telah dikenalpasti yang dikategorikan sebagai: i) kelebihan video sebagai alat mengajar, ii) halangan dalam menggunakan video sebagai alat pengajaran; dan iii) cadangan dalam menggunakan video sebagai alat pengajaran. Kesimpulannya, kajian ini menunjukkan bahawa paramedik mencadangkan pembelajaran melalui video adalah satu keadah yang berpotensi untuk mempelajari skil kecemasan. Walau bagaimanapun, mereka mencadangkan bahasa pengajaran dalam video adalah terutamanya dalam bahasa ibunda mereka supaya ini dapat memudahkan kefahaman.</p>\r\n', 1),
(6648, 6639, 'Latar belakang: Polip hidung jenis eosinopil sangat banyak di negeri Barat. Tujuan utama kajian ini adakah untuk mengetahui bahawa histologi polip hidung di kalangan rakyak Malaysia adakah sama dengan polip populasi Barat. \r\n\r\nMetodologi: Data-data seramai 122 pesakit yang mengidap kronik rinosinusitis dan terdapat polip di dalam hidung mereka telah dikenalpasti. Kepingan histologi untuk sampel polyp hidung telah dikumpulkan. Mikroskop digunakan untuk mengukur bilangan sel-sel inflamasi yang terlibat.  \r\n\r\nKeputusan: Kecenderungan sel eosinopil didapati sebanyak 32.8% dan bukan eosinopil adalah sebanyak 67.2%. Jenis histologi polip hidung menunjukkan sangat signifikan di antara kaum (x² = 8.322; p < 0.05). Sebanyak 78.9% polip hidung dari kaum Cina adalah bukan eosinopil, sementara kaum Melayu dan India menunjukkan 71.9% dan 40.7% setiap kaum. \r\n\r\nKonklusi: Kajian ini menunjukkan kecenderungan polip hidung di dalam populasi Malaysia adalah bukan eosinopil dan ianya sangat signifikan bagi kaum Cina.\r\n', 1),
(7009, 7000, '<p>\r\n	Dengkuran yang nyaring disebabkan oleh sista subglotic adalah keadaan yang jarang berlaku kepada bayi tetapi ia boleh membawa maut jika tidak dirawat dengan betul kerana ia boleh menyebabkan terhalang saluran pernafasan dan mengakibatkan komplikasi yang serius. Kepelbagaian simptom-simptom dan tanda-tanda klinikal menjadikan cabaran yang sangat besar dalam mendiagnosis sista subglotic ini. Manifestasi klinikal yang berbeza ini adalah kerana ia bergantung kepada saiz dan lokasi sista. Kami melaporkan berkenaan kes seorang bayi perempuan berumur 6 bulan yang mempunyai sista subglotic tetapi telah dirawat sebagai penyakit &ldquo;croup&rdquo; sehingga beliau mengalami kegagalan sistem pernafasan di mana proses intubasi menghadapi cabaran yang sangat besar. Oleh itu, kes ini menekankan bahawa para pegawai perubatan perlu memikirkan diagnosis sista subglotic pada mana-mana bayi pra-matang yang datang ke kecemasan dengan bunyi dengkuran yang nyaring serta pernah melalui proses intubasi dahulu. Ini kerana jika diagnosis telah dibuat, rawatan yang sewajarnya dapat diberikan untuk mengelakkan kecacatan dan kematian yang boleh dielakkan.</p>\r\n', 1),
(7032, 7023, 'Analgesia kawalan pesakit (PCA) melalui pam infusi membolehkan pesakit untuk mengawal analgesia mereka sendiri. Tujuan kajian ini adalah untuk menilai kesan program pendidikan analgesia kawalan pesakit (PCA) dari segi kesakitan pos operatif dan kepuasan pesakit dengan pengurusan kesakitan selepas pembedahan ortopedik. Kajian bentuk kuasi-eksperimen pra dan pos reka intervensi dengan pelaksanaan program pendidikan pesakit di PCA yang diberikan kepada 54 responden secara individu yang memenuhi kriteria kemasukan sebagai kumpulan interventasi. Kumpulan kawalan diberi taklimat PCA konvensional dari protokol Acute Pain Service. Tahap kesakitan diukur pada dua jam, enam jam dan 24 jam selepas pembedahan pada pra dan ujian pos dengan penggunaan "Revised American Pain Society Patient Outcome Questionnaire (APS-POQ-R)"  Hasil kajian menunjukkan perbezaan responden tahap skor kesakitan oleh responden kawalan   "median" di dua jam, enam jam dan 24 jam selepas pembedahan,  ialah 7.00 ( IQR = 3.00) , 5.00 ( IQR = 2.00) dan 3.00 ( IQR = 2.00) ; kumpulan interventasi  di dua jam, enam jam dan 24 jam selepas pembedahan adalah 6.00 ( IQR = 2.00) , 3.00 ( IQR = 1.00 ) dan 1.00 ( IQR = 1.00). Terdapat perbezaan yang signifikan dalam median skor kesakitan antara kumpulan interventasi dan  kumpulan kawalan bagi dua (U = 142.0 , p < 0.05) , enam ( U = 150.50 , p < 0.05) dan 24 ( U = 120.00 , p < 0.05) jam selepas pembedahan . Terdapat perbezaan statistik yang signifikan (p < 0.05 ) dalam median sakit tahap pesakit di semua peringkat kesakitan biasa, kesakitan teruk, dan kesakitan yang amat teruk antara interventasi dan kumpulan kawalan (kesakitan biasa, U = 219,50 , p < 0.05 , kesakitan teruk , U = 117.0 , p < 0.05 ; kesakitan yang amat teruk, U = 49.0 , p < 0.05). Kesimpulannya, pesakit yang menerima program pendidikan berstruktur pra-pembedahan menunjukkan peningkatan dalam menguruskan kesakitan selepas pembedahan dan kepuasan pada PCA selepas pembedahan ortopedik. Pengukuhan program pendidikan berstruktur adalah penting dalam mencapai jagarawatan optimum pesakit.', 1),
(7245, 7236, '<p>\r\n	Hipoglisemia, iaitu paras glukosa darah yang rendah, adalah masalah yang lazim berlaku di kalangan pesakit diabetes. Pengukuran gejala hipoglisemia dalam penyelidikan memerlukan alat kajian yang sahih. Hypoglycaemia Symptom Rating Questionnaire (HypoSRQ) adalah soal selidik keadaan hipoglisemia yang telah dibentuk dan melalui proses validasi dalam versi Bahasa Inggeris di United Kingdom oleh Health Psychology Research Ltd (HPR). Kajian ini bertujuan untuk menghasilkan versi HypoSRQ dalam Bahasa Melayu dan Bahasa Inggeris yang bersesuaian dengan bahasa dan budaya Malaysia. Kajian ini dijalankan dari Januari 2014 sehingga Jun 2015. Proses utama yang dijalankan adalah penterjemahan, pengubahsuaian dengan penilaian pakar psikologi, maklum balas kognitif (cognitive debriefing). Proses penterjemahan melibatkan dua versi terjemahan ke hadapan, iaitu versi Bahasa Inggeris ke Bahasa Melayu, dan dua terjemahan berbalik, iaitu versi Bahasa Melayu ke Bahasa Inggeris. Jawatankuasa berbincang untuk memilih istilah bahasa yang paling tepat berpandukan laporan penilaian pakar psikologi. Versi Bahasa Melayu dan Bahasa Inggeris yang telah diterima diuji melalui proses maklum balas kognitif di kalangan pesakit kencing manis. Proses ini dilakukan dengan kerjasama rapat bersama HPR untuk memastikan ketepatan maksud penterjemahannya. Hasil kajian dan maklum balas kognitif telah membuktikan kesahihan versi Bahasa Melayu dari adaptasi versi Bahasa Inggeris. Kajian ukuran psikometrik disyorkan bagi menambahkan kesahihan versi-versi tersebut bagi kegunaan penyelidikan tempatan.</p>\r\n', 1),
(7248, 7239, '<p>\r\n	Polip hidung jenis eosinofil sangat banyak di negeri Barat. Tujuan utama kajian ini adakah untuk mengetahui bahawa histologi polip hidung di kalangan rakyat Malaysia adakah sama dengan polip populasi Barat. Data seramai 122 pesakit yang mengidap kronik rinosinusitis dan terdapat polip di dalam hidung mereka telah dikenalpasti. Kepingan histologi untuk sampel polyp hidung telah dikumpulkan. Mikroskop digunakan untuk mengukur bilangan sel-sel inflamasi yang terlibat. Kecenderungan sel eosinofil didapati sebanyak 32.8% dan bukan eosinofil adalah sebanyak 67.2%. Jenis histologi polip hidung menunjukkan sangat signifikan di antara kaum (x&sup2; = 8.322; p &lt; 0.05). Sebanyak 78.9% polip hidung dari kaum Cina adalah bukan eosinofil, sementara kaum Melayu dan India menunjukkan 71.9% dan 40.7% setiap kaum. Kajian ini menunjukkan kecenderungan polip hidung di dalam populasi Malaysia adalah bukan jenis eosinofil dan ianya sangat signifikan bagi kaum Cina.</p>\r\n', 1),
(7253, 7244, NULL, NULL),
(7257, 7248, '<p>\r\n	-</p>\r\n', 1),
(7261, 7252, '<p>\r\n	Keperluan keluarga pesakit yang sedang menunggu waris di Unit Rawatan Rapi perlu dipenuhi oleh kakitangan kesihatan kerana ini membantu untuk meningkatkan kualiti hidup pesakit. Tujuan kajian ini adalah untuk mengenal pasti keperluan ahli keluarga pesakit yang sedang menunggu waris yang sedang dirawat di Unit Rawatan Rapi. Satu kajian keratan rentas telah dijalankan ke atas 109 ahli keluarga pesakit yang sedang dirawat di Unit Rawatan Rapi dan Unit Rawatan Koronari, Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Soalan kajian yang telah diubah suai daripada Critical Care Family Needs Inventory (CCFNI) telah digunapakai dalam kajian ini. Soalan kajian terdiri daripada 5 domain keperluan ahli keluarga: Maklumat, Berada hampir dengan pesakit, Jaminan, Keselesaan dan Sokongan. Dapatan kajian menunjukkan bahawa keperluan jaminan (3.77 &plusmn; 0.306) adalah yang tertinggi. Ini diikuti dengan keperluan mendapatkan maklumat (3.62 &plusmn; 0.379), berada hampir dengan pesakit (3.60 &plusmn; 0.415), keperluan sokongan (3.57 &plusmn; 0.477) dan keperluan keselesaan (3.55 &plusmn; 0.586). Terdapat hubungan yang signifikan di antara hubungan responden dengan keperluan keluarga berada berdekatan dengan pesakit (p = 0.013). Kajian ini menunjukkan terdapat hubungan yang signifikan antara pendapatan bulanan keluarga dengan keperluan keselesaan dan sokongan, (p = 0.033) dan (p = 0.004). Terdapat juga hubungan yang signifikan antara jantina dengan keperluan keselesaan (p = 0.013). Keperluan menerima maklumat, berada berdekatan dengan pesakit, jaminan, keselesaan dan sokongan adalah amat penting kepada ahli keluarga pesakit. Pakej pendidikan dan sentiasa memberi informasi terkini perlu diberi penekanan bagi meningkatkan keperluan keluarga pesakit kritikal dalam persekitaran penjagaan kritikal.</p>\r\n', 1),
(7268, 7259, '<p>\r\n	Midazolam adalah salah satu ubat yang paling biasa digunakan sebagai ubat pelali di Jabatan Kecemasan (ED). Ini adalah satu kajian retrospektif yang dijalankan ke atas 380 pesakit dari Disember 2012 hingga Mei 2014 di ED, Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Objektifnya adalah untuk mendapatkan kekerapan kesan sampingan dan korelasi kepada pelbagai faktor iaitu sosio-demografi, kumpulan umur dan penyakit-penyakit utama. Daripada 380 pesakit, 35 pesakit mempunyai kesan sampingan (20 pesakit dengan midazolam sahaja, 15 pesakit dengan kombinasi ubat-ubatan). Umur purata adalah 42 tahun dan dos purata midazolam adalah 3.5mg. Ubat lain yang paling biasa di gabung adalah fentanyl. Kadar komplikasi keseluruhan midazolam adalah 5.3%. Kesan sampingan yang paling biasa yang dicatatkan adalah somnolen (1.6%). Kesan sampingan yang lain adalah reaksi alergi kulit (1.1%), muntah (0.8%), sakit kepala (0.8%) dan tekanan darah rendah (0.5%). Tidak ada hubungan yang signifikan antara faktor-faktor sosio-demografi dan kombinasi ubat-ubatan dengan kesan sampingan midazolam pada pesakit. Kesimpulan daripada kajian ini ialah midazolam adalah dadah yang sangat selamat kerana ia tiada kesan sampingan yang mengancam nyawa. Terdapat kemungkinan bahawa kesan-kesan sampingan direkodkan boleh disebabkan oleh faktor-faktor lain seperti kecederaan atau penyakit dan kombinasi dengan ubat-ubatan lain.</p>\r\n', 1),
(7270, 7261, '<p>\r\n	Objektif kajian ini adalah untuk mengenalpasti faktor-faktor yang berkaitan dengan kehadiran semula pada peringkat awal pesakit asma yang didiscajkan daripada Jabatan Kecemasan. Ia adalah satu kajian kohort retrospektif untuk pesakit-pesakit yang berumur 12 tahun atau lebih dalam tempoh 1 bulan yang telah dirawat untuk asma akut dan didiscajkan daripada Jabatan Kecemasan, Hospital Umum Sarawak. Seramai 397 pesakit memenuhi kriteria sampel dan daripada jumlah ini, 13.9% telah hadir semula dalam tempoh 2 minggu. Daripada kes-kes kehadiran semula, 9.1% dimasukkan ke wad. Kadar perskripsi kortikosteroid oral adalahi rendah (24.9 %) dan melarikan diri daripada hospital adalah tinggi (25.1 %). Pesakit yang telah melarikan diri dari jabatan kecemasan dan jangkitan mereka berkadar terus dengan ketibaan semula ke Jabatan Kecemasan.</p>\r\n', 1),
(7273, 7264, '<p>\r\n	Pesakit yang pulang tanpa nasihat perubatan (DAMA) terdedah kepada akibat yang boleh mengancam nyawa. Dengan memahami faktor-faktor yang berkaitan dengan DAMA, pusat-pusat penjagaan kesihatan boleh membina strategi untuk membantu pesakit menerima rawatan perubatan yang optimum dan mengelakkan komplikasi yang lebih buruk. Objektif kajian ini adalah untuk menentukan faktor-faktor yang berkaitan dengan DAMA dari Jabatan Perubatan Kecemasan, Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Kajian ini telah dijalankan dalam tempoh 4 bulan. Bagi setiap DAMA, dua pesakit lain yang dimasukkan ke wad pada hari sama telah dipilih secara rawak sebagai kumpulan kawalan. Data telah dikumpulkan melalui borang soal selidik setelah mendapatkan persetujuan pesakit. Pesakit telah dihubungi dalam masa dua minggu selepas DAMA. Dari 93 pesakit, kumpulan DAMA mempunyai 31 pesakit dan kumpulan kawalan mempunyai 62 pesakit. Kaedah pembayaran adalah faktor ketara bagi pesakit menambil DAMA (OR 3.17 95% CI 1.29-7.98; p = 0.01). Kebarangkalian DAMA bagi pesakit yang membayar kos perawatan sendiri adalah tiga kali lebih tinggi daripada mereka yang mempunyai surat penjamin daripada majikan atau syarikat insurans. Faktor-faktor lain yang mempengaruhi DAMA adalah keluarga (OR 4.08 95% CI 1.09-15.26; p = 0.03) dan kerja (OR 3.83 95% CI 1.13-12.94; p = 0.03). Sebanyak 19.4% pesakit mengambil DAMA berikutan gejala kesakitan berkurangan. Sebanyak 80.6% pesakit telah dimasukkan semula ke hospital dalam tempoh dua minggu setelah DAMA. Kaedah pembayaran mempengaruhi keputusan untuk DAMA. Kemudahan pembayaran yuran secara berkala, perkhidmatan kebajikan sosial, bantuan dana dari organisasi bukan kerajaan dan skim dasar kesihatan kebangsaan boleh mengurangkan DAMA.</p>\r\n', 1),
(7280, 7271, '<p>\r\n	Mangsa serangan jantung di luar hospital (OHCA) memerlukan bantuan bantu mula jantung (CPR) dan defibrilasi dengan segera sebelum ketibaan di Jabatan Kecemasan. Tindakan bantu mula CPR ini boleh meningkatkan peluang untuk terus hidup daripada serangan jantung secara tiba-tiba. Objektif utama kajian ini adalah untuk mengenal pasti faktor-faktor yang mempengaruhi hasil dari tindakan untuk melakukan CPR kepada mangsa OHCA di Jabatan Kecemasan, Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Objektif kedua adalah untuk menentukan jumlah CPR yang dimulakan oleh orang ramai sebelum ambulans tiba. Kajian rentas ini adalah selama satu tahun. Pesakit OHCA telah dikenal pasti dari buku log CPR. Rekod perubatan pesakit telah digunakan untuk mendapatkan butir-butir bantuan kecemasan yang dilakukan. Faktor-faktor yang dicatatkan termasuk: sebab pengsan, permulaan CPR di tempat kejadian, penggunaan defibrillator automatik (AED), jenis pengangkutan dan kejadian pulangan peredaran darah secara spontan (ROSC). Data dianalisis secara kategori menggunakan ujian chi-square dan Fisher. Sembilan pesakit daripada 98 menerima CPR dari tempat kejadian. Tiga pesakit mencapai ROSC. Faktor jantina mempunyai kaitan yang ketara dengan ROSC (p = 0.015). Lebih ramai pesakit yang menerima bantuan awal CPR mencapai ROSC berbanding dengan mereka yang lewat menerima CPR. Kadar bantuan awal CPR dan penggunaan AED oleh orang ramai masih rendah. Jantina perempuan mempunyai pengaruh yang positif ke atas ROSC. Usaha-usaha diperlukan untuk meningkatkan kesedaran dan penglibatan orang awam untuk memulakan bantuan awal CPR sebelum ketibaan perkhidmatan ambulans.</p>\r\n', 1),
(7283, 7274, '<p>\r\n	Penggunaan teknologi informasi merupakan salah satu kaedah pengajaran popular dalam bidang perubatan. Salah satu kaedah yang digunakan ialah pembelajaran berasaskan video (PBV). Pengunaan video sebagai bahan bantu pengajaran bagi pembelajaran kemahiran kecemasan bukan sesuatu yang baru. Namun, kajian mengenai persepsi penggunaan PBV ini masih kurang. Kajian kualitatif ini melibatkan empat perbincangan kumpulan fokus setelah peserta mengikuti sesi pembelajaran skil melalui PBV. Seramai 20 orang paramedik dibahagikan kepada empat kumpulan fokus. Mereka terlibat dalam perbincangan setelah mengikuti PBV. Hasil kajian ini menunjukkan bahawa terdapat tiga tema utama yang dikategorikan sebagai: i) kelebihan video sebagai alat bantu mengajar, ii) halangan dalam menggunakan video sebagai alat bantu pengajaran; dan iii) cadangan dalam menggunakan video sebagai alat pengajaran. Kajian ini menunjukkan bahawa paramedik yang terlibat merasakan video berpotensi sebagai satu kaedah dalam mempelajari kemahiran kecemasan. Walau bagaimanapun, mereka mencadangkan bahasa ibunda digunakan sebagai bahasa pengantara dalam pengajaran berasaskan video ini. Ini menunjukkan bahawa penggunaan Bahasa Inggeris dalam PBV mempunyai kelemahan untuk memahami kandungan pembelajaran.</p>\r\n', 1),
(7291, 7282, '<p>\r\n	Polip hidung jenis eosinofil biasa terjadi dalam populasi Barat. Tujuan utama kajian ini adalah untuk menentukan jenis histologi polip hidung dalam kalangan kumpulan etnik di Malaysia. Data demografi 122 pesakit yang mengidap kronik rinosinusitis dan mempunyai polip di dalam hidung mereka telah dikenalpasti dan direkod. Kepingan histologi untuk sampel polyp hidung telah dikumpulkan. Bilangan eosinofil dan bukan eosinofil serta purata sel-sel inflamasi yang terlibat dikira. Kecenderungan sel eosinofil didapati sebanyak 32.8% dan bukan eosinofil adalah sebanyak 67.2%. Jenis histologi polip hidung menunjukkan sangat signifikan di antara kaum (x&sup2; = 8.322; p &lt; 0.05). Sebanyak 78.9% polip hidung dari kaum Cina adalah bukan eosinofil, sementara peratusan bagi kaum Melayu dan India masing-masing adalah 71.9% dan 40.7%. Kajian ini menunjukkan kecenderungan polip hidung di dalam populasi Malaysia adalah jenis bukan eosinofil dan ianya sangat signifikan bagi kaum Cina.</p>\r\n', 1),
(7293, 7284, 'Seorang lelaki berusia 18 tahun itu yang tiba di jabatan kecemasan dengan sakit dada sebelah kanan dan sesak nafas. X-ray dada ditafsir sebagai pneumothorax spontan. Aspirasi jarum telah dijalankan tetapi keadaan pesakit merosot. Kemudian tiub dada torakostomi dilakukan. Selepas itu berkata pendarahan berlaku di dalam thorax. Keadaan pesakit merosot. CT toraks telah dilakukan dengan segera dan menunjukkan bahawa terdapat hemopneumothorax  dengan banyak bullae. Pesakit telah ditolak ke bilik pembedahan untuk thorakotomi. Pelajaran yang boleh dipelajari daripada kes ini adalah bahawa bullae emphysema gergasi boleh menyamar sebagai pneumotoraks dan perhatian lebih perlu diamalkan sebelum melaksanakan thoracocentesis.', 1),
(7295, 7286, '<p>\r\n	Analgesia kawalan pesakit (PCA) melalui pam infusi membolehkan pesakit untuk mengawal analgesia mereka sendiri. Tujuan kajian ini adalah untuk menilai kesan program pendidikan analgesia kawalan pesakit (PCA) dari segi kesakitan pos pembedahan dan kepuasan pesakit dengan PCA selepas pembedahan ortopedik. Kajian bentuk kuasi-eksperimen pra dan pos rekaintervensi dengan pelaksanaan program pendidikan pesakit di PCA yang diberikan kepada 54 responden. Kumpulan kawalan diberi taklimat PCA konvensional dari protokol Acute Pain Service. Tahap kesakitan diukur pada 2 jam, 6 jam dan 24 jam selepas pembedahan dengan menggunakan ujian pra dan pos dari pada pra dan ujian pos dengan penggunaan &ldquo;Revised American Pain Society Patient Outcome Questionnaire (APS-POQ-R)&rdquo;. Hasil kajian menunjukkan perbezaan median respon tahap skor kesakitan oleh responden kawalan pada 2 jam, 6 jam dan 24 jam selepas pembedahan, ialah 7.00 (IQR = 3.00), 5.00 (IQR = 2.00) dan 3.00 (IQR = 2.00); kumpulan interventasi pada 2 jam, 6 jam dan 24 jam selepas pembedahan adalah 6.00 (IQR = 2.00), 3.00 (IQR = 1.00) dan 1.00 (IQR = 1.00). Terdapat perbezaan yang signifikan dalam median skor kesakitan antara kumpulan interventasi dan kumpulan kawalan bagi 2 (U = 142.0, p &lt; 0.05) , 6 (U = 150.50 , p &lt; 0.05) dan 24 (U = 120.00, p &lt; 0.05) jam selepas pembedahan. Terdapat perbezaan statistik yang signifikan (p &lt;0.05) dalam median tahap sakit pesakit di semua peringkat kesakitan biasa, kesakitan teruk, dan kesakitan yang amat teruk antara kumpulan interventasi dan kumpulan kawalan (kesakitan biasa, U = 219.50, p &lt; 0.05, kesakitan teruk , U = 117.0, p &lt; 0.05; kesakitan yang amat teruk, U = 49.0, p &lt; 0.05). Kesimpulannya, pesakit yang menerima program pendidikan berstruktur pra-pembedahan menunjukkan peningkatan dalam menguruskan kesakitan selepas pembedahan dan kepuasan pada PCA selepas pembedahan ortopedik.</p>\r\n', 1),
(7297, 7288, '<p>\r\n	Hypoglycaemia Symptom Rating Questionnaire (HypoSRQ) merupakan soalan kaji selidik bagi mengukur simptom hipoglysaemia dan ia berpotensi untuk kegunaan penyelidikan tempatan. Namun, ia memerlukan adaptasi dan validasi untuk kegunaan tempatan. Kajian ini melaporkan proses dan hasil keputusan adaptasi rentas budaya dan validasi linguistik bagi HypoSRQ dalam Bahasa Melayu dan Bahasa Inggeris di dalam situasi tempatan. HypoSRQ telah melalui proses penterjemahan berbalik dan adaptasi dengan sokongan penterjemah profesional dan pakar psikologi klinikal. Maklum balas kognitif (cognitive debriefing) dilakukan di kalangan pesakit diabetes mellitus jenis 1 dan 2 dengan latar belakang sosiodemografi yang berlainan. Perbincangan bersama pembangun HypoSRQ asal, iaitu Health Psychology Research Ltd (HPR) dilakukan untuk menentukan versi terbaik bagi kegunaan tempatan. Versi akhir disemak dan diformatkan. Maklum balas kognitif melibatkan 7 orang pesakit bagi HypoSRQ-My (versi bahasa Melayu) dan 5 orang pesakit bagi HypoSRQ-EMy (versi bahasa Inggeris). Penterjemahan langsung secara literal kepada bahasa Melayu tidak sesuai disebabkan istilah teknikal yang sukar difahami oleh masyarakat umum. Pindaan dibuat berdasarkan penemuan daripada proses maklum balas kognitif. Peserta kajian mendapati bahawa soal selidik agak mudah untuk difahami selepas pindaan. HypoSRQ-My (versi bahasa Melayu) dan HypoSRQ-EMy (versi bahasa Inggeris) mudah difahami oleh masyarakat tempatan. Soal selidik ini akan melalui penilaian psikometrik untuk kegunaan tempatan pada masa akan datang.</p>\r\n', 1),
(7300, 7291, '<p>\r\n	Laporan kes ini mengetengahkan kaedah penstablian terapi elektrokonvulsif (ECT) yang cepat bagi pesakit kemurungan major (MDD) yang mempunyai indeks jisim berat badan (BMI) tersangat rendah. Kami melaporkan seorang suri rumah wanita berumur 55 tahun dengan masalah penyakit hipertiroid yang berada di dalam status eutiroid mengalami masalah kemurungan major dengan ciri khusus psikosis kongruen akibat daripada kematian suami pesakit. Paras BMI beliau hanyalah pada tahap 11 kg/m2 disebabkan oleh anoreksia dan pesakit berkenderungan untuk membunuh diri. Preskripsi pemberian nutrisi secara parenteral sepenuhnya diberi dan rawatan ECT telah dilaksanakan untuk rawatan penyakit kemurungan secara penstabilan yang cepat di samping rawatan oral menggunakan ubat escitalopram 15 mg setiap malam. Penyembuhan sepenuhnya tercapai selepas rawatan terapi sebanyak sembilan kali ECT dan berat badan naik secara sihat di dalam wad. Pesakit telah dilepaskan pada tahap BMI sebanyak 13 kg/m2 dengan selera makan yang baik. Secara kesimpulannya, rawatan ECT adalah selamat untuk pesakit kemurungan major yang mempunyai paras indeks jisim berat badan sangat rendah.</p>\r\n', 1),
(7303, 7294, '<p>\r\n	Vaginismus adalah disfungsi seksual yang disebabkan oleh pengecutan otot-otot faraj yang menyebabkan kemasukan zakar semasa hubungan seks amat sukar, atau mustahil berlaku. Ia sering diiringi tekanan perasaan yang ketara. Di dalam kebanyakan kes, ia berkait-rapat dengan perasaan ketakutan melampau yang meneruskan hayat dan sifat penyakit ini. Kami melaporkan kes vaginismus primer yang menyebabkan komplikasi psikiatri jenis kerisauan-kemurungan lalu mencadangkan pendekatan perawatan holistik yang merangkumi pendekatan tingkah-laku dan psikofarmakologi. Rawatan permasalahan psikiatri yang berkait-rapat dengan vaginismus harus terlebih dahulu ditangani, secara baik bersama-sama rawatan kepada vaginismus. Kombinasi bersepadu pendekatan tingkah-laku, psikologi dan farmakologi amat penting bagi memastikan prognosis rawatan adalah baik.</p>\r\n', 1),
(7306, 7297, '<p>\r\n	Dengkuran nyaring akibat sista subglotik adalah keadaan yang jarang berlaku kepada bayi. Namun, ia boleh membawa maut jika tidak dirawat dengan betul kerana boleh menyebabkan terhalangnya saluran pernafasan dan mengakibatkan komplikasi yang serius. Kepelbagaian simptom dan tanda klinikal merupakan cabaran yang sangat besar dalam mendiagnosis sista subglotik ini. Manifestasi klinikal berbeza kerana ia bergantung kepada saiz dan lokasi sista. Kami melaporkan kes seorang bayi perempuan berumur 6 bulan yang mempunyai sista subglotik tetapi telah dirawat sebagai penyakit &ldquo;croup&rdquo;sehingga beliau mengalami kegagalan sistem pernafasan dan kesukaran proses intubasi. Oleh itu, adalah ditekankan supaya para pegawai perubatan perlu memikirkan diagnosis sista subglotik pada bayi-bayi pra-matang yang datang ke kecemasan dengan bunyi dengkuran yang nyaring serta pernah melalui proses intubasi. Selepas diagnosis yang tepat dibuat, rawatan yang sewajarnya dapat diberikan untuk mengelakkan kecacatan dan kematian.</p>\r\n', 1),
(7309, 7300, '<p>\r\n	Perubatan tradisional atau herba secara umumnya dianggap sebagai selamat kerana ia diperolehi dari sumber asli atau tumbuhan. Kebelakangan ini, terdapat peningkatan bilangan laporan kes alahan akibat penggunaan perubatan tradisional termasuk tindak balas pada kulit. Reaksi alahan pada kulit, lazimnya akan merangkumi kawasan kulit yang bersentuhan dengan ubat. Kami melaporkan kes seorang wanita berumur 65 tahun yang mengalami kemerahan, rasa terbakar dan bengkak selepas menggunakan minyak tradisional Cina. Laporan ini adalah sebagai garis panduan untuk mengenalpasti dan mengurus masaalah alahan ubat sapu pada kulit. Adalah diharapkan kes ini memberi kesedaran bahawa perubatan tradisional tidak bebas dari reaksi alahan. Para doktor perlu arif tentang kesan alahan terhadap perubatan tradisional dan perlu mengambil langkah berjaga-jaga, sama seperti yang diamalkan untuk rawatan konvensional.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(7313, 7304, '<p>\r\n	Kedudukan pesakit di atas meja pembedahan adalah penting dalam memastikan keselesaan pesakit, maruah pesakit dan akses yang baik kepada pakar bedah. Kedudukan yang tepat membantu dalam mengurangkan risiko kecederaan kepada pesakit. Beg udara rekaan sendiri boleh dibuat dengan bekalan perubatan dikitar semula di dewan pembedahan. Peralatan rekaan ini boleh menggantikan beg pasir konvensional untuk pembedahan kepala dan leher. Ia melibatkan penggunaan botol pengairan yang kosong, injap dan tiub sedutan. Bentuk beg udara boleh dilaraskan dengan mudah dengan mengubah isipadu udara di dalam beg. Penggunaan beg yang berisi udara membolehkan beg mengubah bentuk dan mengacukan permukaannya berdasarkan kelengkungan permukaan luar pesakit. Ini mengurangkan risiko tekanan pada kulit teruamanya dalam pembedahan yang berpanjangan. Kesimpulannya, kaedah ini adalah suatu teknik bijak, inovatif dan menjimatkan perbelanjaan kewangan dengan menggunakan bahan kitar semula.</p>\r\n', 1),
(7318, 7309, 'BM version: \r\nKami membentangkan kes berkenaan tentang lelaki yang mempunyai sejarah bengkak pada bahagian “parotid” kanan selama setahun. Klinikal diagnosi sebagai kanser parotid. Pengimejan diagnosi adalah ketumbuhan di bahagian ‘ mastikator” dan merebak kebahagian ‘infratemporal” and pathologi diagnosi sebagai “ Giant Cell Tumor”. Kes ini membincangkan tumor yang tipikal tetapi dikawasan yang tidak tipikal.\r\n', 1),
(7319, 7310, '<p>\r\n	Pertambahan dalam aplikasi radiasi serta kurangnya kesedaran pekerja untuk meggunakan peralatan keselamatan menyebabkan risiko dedahan radiasi kepada mata meningkat. Kajian ini bertujuan untuk menentukan status penglihatan warna serta hubungannya dengan tempoh bekerja dikalangan kakitangan yang bekerja dengan radiasi di sebuah hospital universiti. Seramai 56 orang pekerja (29 terdedah secara terus (RS) dan 27 tidak terdedah secara terus (NRS)) terlibat dalam kajian ini dan 30 orang lagi digunakan sebagai subjek kawalan. Akuiti visual diukur mengguakan carta Snellen. Penglihatan warna diuji menggunakan plat Ishihara, panel D15 dan FM 100hue. Keputusan kajian dianalisa menggunakan ANOVA sehala. Julat umur subjek adalah 22 hingga 44 tahun. Purata VA untuk semua subjek adalah 6/6. Lebih 50% subjek mempunyai defek penglihatan warna, 3.45% RS gagal ujian D15, 37.93% gagal ujian FM100hue, 7.4%NRS gagal ujian D15, 55.56% gagal ujian FM100hue. Analisa menunjukkan perbezaan signifikan dalam skor total error antara RS (77.10 &plusmn; 6.05) dan NRS (84.89 &plusmn; 7.76) berbanding kawalan (41.47 &plusmn; 3.10) [ANOVA F(2,83)=16.084, p=0.00]. Tiada hubungan didapati antara tempoh bekerja dengan keterukan penglihatan warna [r(104)= -0.123, p&gt;0.05]. Kajian ini menyimpulkan kebanyakan kakitangan kajian mempunyai defek penglihatan warna tetapi ianya tidak berkait dengan tempoh bekerja. Langkah keselamatan perlu diambil untuk memperbaiki keadaan tersebut.</p>\r\n', 1),
(7328, 7319, NULL, NULL),
(7339, 7330, NULL, NULL),
(7363, 7354, '<p>\r\n	Mutasi telah di kenal pasti berlaku sebelum pembentukan pelbagai jenis kanser. Mutasi di dalam gen p53 boleh di jumpai dalam kanser payudara. Objektif kajian ini adalah untuk menentukan korelasi antara protein p53 dengan wanita dalam usia pra-menopaus. Sebanyak 111 tisu kanser payudara telah dikaji untuk ekspresi protein p53 oleh (Immunohistochemistry)IHC. Hasil kajian mendapati majoriti 36.9.% (n = 41/111) wanita berumur 41 tahun ke atas yang menghidap kanser payudara ekspress lebih p53 protein positif (+) dan hanya 2.7% (n = 3/111) bawah 41 tahun ekspress protein p53 positif (+) berlebihan. Walau bagaimanapun, ujian Fishers Exact menunjukkan bahawa, terdapat hubungan yang tidak signifikan antara kumpulan umur peserta dengan kategori proteein p53 (&chi;2 (1) = 0.780; p = 0.522); Cramer&rsquo;s V = 0.084; p = 0.377). Anggaran risiko menunjukkan kebarangkalian ekspresi protein p53 (+) yang melampau dalam kumpulan yang berusia bawah daripada 41 tahun adalah 0.66 kali lebih kurang berbanding kumpulan wanita yang berusia 41 tahun keatas. Kesimpulannya dengan atau tanpa ekspresi protein p53 yang melampau, wanita yang berusia lebih daripada 41 tahun mempunyai risiko yang lebih besar untuk menghidap kanser payudara.</p>\r\n', 1),
(13150, 13141, '<p>\r\n	Kecederaan buku lali adalah kejadian yang biasa berlaku dalam mana-mana aktiviti sukan. Tujuan utama kajian adalah untuk menjelaskan anatomi buku lali, mekanisma kecederaan yang berkaitan dengan aktiviti sukan, keabnormalan secara kongenital atau variasi anatomi yang berkaitan dengan kecederaan buku lali serta perbincangan rawatan secara efektif. Suatu tinjauan literatur telah dijalankan untuk mengetahui kecederaan buku lali yang berlaku akibat daripada pelbagai aktiviti sukan. Kami mendokumenkan semua sukan yang melibatkan kecederaan pada sendi buku lali. Anatomi pelbagai struktur bahagian tapak kaki dan keterlibatannya dalam kecederaan dibincangkan dengan panjang lebar. Pengetahuan anatomi tentang kecederaan buku lali boleh memberi manfaat untuk diagnosis akan datang dan bagi tujuan rawatan.</p>\r\n', 1),
(13164, 13155, 'Rawatan konvensional laser argon menyebabkan ketebalan sementara pada lapisan saraf retina (RNFL). Tetapi kesan dari laser ‘pattern scanning laser’ (PASCAL) tidak banyak diketahui. Kajian ini dilakukan untuk membandingkan kesan selepas knovensional laser argon dibandingkan dengan PASCAL pada pesakit yag mengalami masalah diabetic retinopati. Sebanyak 32 subjek telah terpilih terlibat dalam kajian ini.  16 pesakit dalam kumpulan argon dan 16 pesakit dalam kumpulan PASCAL. Pesakit terlibat diambil dari klinik mata , Pusat Perubatan Universiti Kebangsaan Malaysia. Pemeriksaan mata yang lengkap dan gambar fundus dilakukan pada awal kajian, selepas 2 bulan dilaser dan 4 bulan dilaser. RNFL diukur menggunakan domain masa tomografi koheren optikal (OCT). Kedua kumpulan mempunyai sama ciri klinikal sebelum rawatan (P= 0.323). RNFL selepas laser argon tidak berubah secaa signifikan dalam semua kuadran pada bulan 2 dan 4 selepas rawatan. Namun, untuk kumpulan PASCAL, perubahan sigifikan telah ditemui pada bulan 4 rawatan pada ‘average RNFL’ dan RNFL ‘inferior’ (P<0.05). Bahagian lain menunjukkan kenaikan ketebalan RNFL tetapi tidak signifikan secara statistik. Sebagai kesimpulan, kenaikan RNFL berlaku pada kedua kumpulan. Pesakit PASCAL mengalami lebih ketebalan. Dengan itu, adalah perlu mengambil kira kejadian laser yang baru berlaku jika mahu menganalisa ketebalan RNFL. ', 1),
(55059, 53295, 'ABSTRAK\r\nPembaziran ubat menimbulkan beban kewangan kepada ekonomi negara, dan juga, implikasi alam sekitar yang berkaitan dengan pelupusan pembaziran. Kajian ini dijalankan di sebuah hospital awam di Brunei dan bertujuan untuk mengukur tahap pembaziran ubat-ubatan dan pelupusannya. Ia adalah satu kajian rentas/silang bahagian yang menggunakan soal selidik direka sendiri untuk para pesakit mengunjungi farmasi pesakit luar di hospital Suri Seri Begawan (SSB); seramai 253 pesakit mengambil bahagian dalam tempoh dua minggu. Terdapat kriteria pengecualian, iaitu pesakit di bawah usia 18 tahun, pesakit mengunjungi Jabatan Kemalangan dan Kecemasan, dan pesakit yang dimasukkan ke hospital. Majoriti peserta dilaporkan mempunyai ubat yang tidak digunakan di rumah (75.1%, n=189). Hampir separuh (54.2%) mempunyai ubat-ubatan yang tidak digunakan dan ia adalah disebabkan oleh peningkatan dalam keadaan perubatan mereka. Kebanyakkan peserta (70.8%) tidak diberi nasihat yang bersesuaian mengenai cara untuk melupuskan ubat-ubatan mereka dengan cara yang betul. Kebanyakkan mereka membuang ubat-ubatan yang tidak digunakan ke dalam tong sampah di rumah (70%), walaupun 50.2% (n=189) mengetahui bahawa membuang ubat-ubatan dalam tong sampah boleh menyebabkan kesan buruk kepada alam sekitar.  Selain itu, didapati bahawa 47.7% daripada peserta akan mengembalikan ubat-ubatan yang tidak digunakan ke farmasi, tetapi, secara praktikal, hanya 18.6% telah melakukannya. Tiada hubungan yang signifikan antara mengetahui kesan-kesan memudaratkan perubatan ke atas alam sekitar dan mengembalikan ubat mereka ke farmasi (p=0.065). Kajian ini menunjukkan bahawa wujud pembaziran ubat di hospital SSB dan pengetahuan serta amalan pesakit terhadap pelupusan yang betul adalah agak lemah. Oleh itu, penggubal dasar kesihatan perlu dimaklumkan mengenainya agar dapat melaksanakan strategi pengurangan pembaziran.\r\n', 1),
(35944, 35934, 'Abstrak\r\n	Plasma cell myeloma telah diketahui menyebabkan pengeluaran  immunoglobulin (Ig) monoklonal yang mengakibatkan rembesan homogen protein monoklonal (komponen M) yang unik. Walau bagaimanapun, terdapat kes-kes melaporkan bahawa ia juga boleh menyebabkan pengeluaran dua klon protein M monoklonal yang berbeza. Walaupun ia agak jarang berlaku dan hanya 2% kes direkodkan daripada semua kes plasma cell myeloma, ciri-ciri klinikal adalah sama dengan mana-mana kes monoklonal tersebut. Berkemungkinan 2 klon protein M yang berbeza ini adalah hasil dari klon yang sama atau dua klon sel monoklonal yang berbeza. Mana-mana pun mekanisme penyakit ini, tindak-balas terhadap rawatan adalah sama dengan kes-kes monoklonal walaupun beberapa laporan menunjukkan rintangan terhadap rawatan boleh berlaku. Di sini, kami melaporkan satu kes plasma cell myeloma yang jarang direkodkan dimana kes ini menunjukkan penghasilan dua klon protein M yang berbeza iaitu IgG (lambda) dan IgA (lambda), ciri-ciri klinikal, ciri-ciri hematologi serta penanda biokimia dan tindak balas kepada rawatan.\r\n\r\n', 1),
(13193, 13184, '<p>\r\n	Tujuan: Untuk menentukan korelasi serum matriks metalloproteinase-3 (MMP-3) dengan aktiviti penyakit, kerosakan sendi dan kecacatan berfungsi pada pesakit dengan Rheumatoid Arthritis (RA). Kaedah: Ini adalah satu kajian kes-kawalan keratan rentas yang melibatkan pesakit RA yang berada di bawah susulan di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Maklumat mengenai ciri-ciri penyakit RA telah diperolehi daripada rekod perubatan dan semua pesakit RA telah dinilai untuk DAS28 (skor aktiviti penyakit berdasarkan 28 sendi) dan Stanford Penilaian Kesihatan Questionnaire (HAQ) 8-item Indeks Upaya (HAQ-DI). The radiograf tangan pesakit RA dijaringkan menggunakan Skor Sharp Modified (MSS). Serum MMP-3 peringkat dari pesakit RA dan kawalan yang sihat diukur menggunakan kaedah ELISA. Keputusan: Seramai 77 pesakit RA dan 18 kawalan sihat telah mendaftar dalam kajian ini. Tahap serum MMP3 adalah lebih tinggi di kalangan pesakit RA (p &lt;0.05). Terdapat hubungan yang signifikan antara serum MMP-3 peringkat dan MSS (r = 0,327) dan HAQ-DI (r = 0,256), kedua-dua p &lt;0.05. Serum tahap MMP min pada pesakit RA dengan hakisan bersama radiografik adalah jauh lebih tinggi daripada pada pesakit tanpa hakisan (p &lt;0.05). Begitu juga, mata pelajaran yang mengalami masalah fungsi penting mis HAQ-DI &ge;1; telah jauh lebih tinggi bermakna tahap MMP3 berbanding pesakit RA tanpa kecacatan yang signifikan (p &lt;0.05). Kepada analisis multivariat, HAQ-DI kekal peramal bebas daripada serum MMP-3 pada pesakit RA. Kesimpulan: Serum MMP3 adalah penanda bio yang berpotensi dan peramal kerosakan sendi radiografi dan kecacatan berfungsi dalam RA.</p>\r\n', 1),
(25691, 25681, NULL, NULL),
(27858, 27848, NULL, NULL),
(27876, 27866, '<p>\r\n	Solar retinopathi merupakan satu penyakit retina yang disebabkan oleh pandangan terhadap cahaya matahari. Diagnosis penyakit ini amat mencabar dan memerlukan sejarah kesihatan dan penyiasatan yang teliti. Laporan kes ini adalah berkenaan seorang pemetik buah-buahan yang berumur 55 tahun yang mengalami kehilangan penglihatan kekal di bahagian tengah medan penglihatan. Diagnosis solar retinopathi telah dibuat berdasarkan kepada sejarah yang diberi oleh pesakit berkenaan, bersama penyiasatan menggunakan pengimejan seperti optical coherence tomography (OCT), infrared (IF) dan fundus autofluorescence (FAF) yang memainkan peranan penting dalam kes ini. Kesimpulannya, solar retinopathi adalah satu penyakit yang tidak boleh dipulihkan. Penyiasatan dengan cara pengimejan memainkan peranan yang penting untuk merangkul diagnosis yang betul. Penyakit ini tidak dapat dipulihkan, justeru itu pemakaian cermin mata hitam adalah disarankan untuk semua pesakit yang sering terdedah kepada cahaya matahari.</p>\r\n', 1),
(27880, 27870, NULL, NULL),
(27934, 27924, 'Kes keracunan plumbum (Pb) yang membimbangkan telah meningkat di beberapa negara tertentu. Terdapat semakin banyak bukti kajian yang menunjukkan bahawa pendedahan kronik terhadap plumbum memainkan peranan besar yang menyebabkan ketidakseimbangan prooksidan: antioksidan di dalam tisu otak dan perubahan histologi otak. Kajian ini dilakukan untuk mengenal pasti kesan agen antioksidan daripada kelapa sawit Malaysia, fraksi kaya tokotrienol (TRF) pada tisu otak tikus eksperimen berikutan keracunan plumbum. Lapan belas (18) tikus Sprague-Dawley jantan, berusia 6 minggu, dibahagikan secara rawak kepada kumpulan kawalan (CTRL) dan kumpulan eksperimen; didedahkan kepada 0.2% w / v plumbum asetat, sebagai kumpulan PB2; dan yang didedahkan kepada 0.2% w / v plumbum asetat bersama pengambilan suplemen TRF (200 mg / kg berat badan) sebagai kumpulan PB2T. Eksperimen ini dijalankan selama 30 hari. Pada akhir kajian, tisu otak telah diambil dan perubahan histopatologi di kawasan hippocampus diperhatikan. Keputusan biokimia seperti aras plumbum, TRF dan malondialdehid (MDA) di dalam tisu otak, dan aras aktiviti superoksida dismutase (SOD) di dalam eritrosit telah ditentukan. Terdapat neuron atipikal berciri apoptosis dan tidak teratur diperhatikan di kawasan hippocampus bagi kumpulan Pb2 yang didedahkan kepada plumbum berbanding kumpulan Pb2T. Parameter- parameter biokimia tisu otak menunjukkan penurunan aras plumbum yang siginifikan (p<0.05) di dalam kumpulan Pb2T berbanding Pb2. Walaupun tiada perbezaan yang signifikan diperolehi (p>0.05) bagi aras MDA, terdapat peningkatan yang signifikan (p<0.05) bagi aktiviti SOD eritrosit dalam PB2T berbanding PB2 dan CTRL. Pengambilan suplemen TRF dilihat dapat membaiki perubahan histopatologi di dalam tisu otak yang disebabkan oleh pendedahan plumbum melalui air minuman dengan mengurangkan pengumpulan plumbum dalam tisu otak tikus eksperimen.', 1),
(28928, 28918, '<p>\r\n	Polypoidal choroidal vasculopathy (PCV) adalah penyakit retina yang dicirikan oleh aneurismal polip dalam vaskulatur koroid. Penyakit PCV kerap berlaku dikalangan populasi yang berpigmen terutama di kalangan orang Asia. Kebanyakan laporan mengenai corak penyakit PCV disekitar Asia, hanya berdasarkan satu bangsa sahaja (e.g. kaum Cina, Jepun, etc). Amat sedikit laporan mengenai corak penyakit ini dalam populasi pelbagai bangsa seperti di Malaysia. Oleh itu, kami bertujuan menggambarkan ciri-ciri demografi, klnikal dan penyiasatan PCV dalam populasi pelbagai bangsa di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Ini adalah kajian retrospektif pada 91 mata dari 86 pesakit PCV, terdiri daripada Cina (65.1%), Melayu (31.4%), India (2.3%) dan Eurasian (1.2%). Semua pesakit telah menjalani pemeriksaan oftalmik dan penyiasatan terperinci. Purata umur pesakit adalah 70.4 tahun, kebanyakannya lelaki (59.3%) dan majoriti mengalami masalah didalam satu mata sahaja (94.1%). Purata penglihatan adalah logMAR 0.78 &plusmn; 0.64. Luka-luka vaskular polypoidal umumnya didapati dalam kawasan makula (86.8%) dan menunjukkan pendarahan terutamanya dibawah lapsian makular (59.3%). Menariknya, beberapa mata (43.9%) menunjukkan &ldquo;drusen&rdquo;. Optical coherence tomografi kebanyakannya menunjukkan perubahan exudative (75.9%) dan hampir semua pesakit (97.7%) mempunyai kehilangan &ldquo;external limiting membrane&rdquo; (ELM) dan IS/OS. Dengan ICG angiografi, majoriti mata didapati ada lebih dari satu polip (82.4%) dan konfigurasi polip yang paling kerap adalah dalam bentuk &#39;kelompok&#39; (58.2%). Kesimpulannya, walaupun secara amnya corak PCV di PPUKM sama dengan corak PCV dipopulasi Asia yang lain, namun beberapa pesakit kami ada &ldquo;drusen&rdquo;. Oleh itu PCV di Malaysia mungkin berlainan dengan popualsi di Asia lain dimana ia mungkin adalah varian degenerasi makula dan tidak &ldquo;idiopathic&rdquo; semata-mata.</p>\r\n', 1),
(29419, 29409, NULL, NULL),
(29420, 29410, 'Tulang metatarsal kelima adalah antara tulang kaki yang paling kerap mengalami patah. Pilihan perawatan sama ada melalui pembedahan ataupun konservatif masih dalam perbincangan. Sistem skor, model pilihan analisis dan sistem klasifikasi telah dibentuk untuk membantu membuat pilihan terbaik sama ada perawatan melalui pembedahan ataupun konservatif. Anatomi yang unik dan komplikasi pembedahan mempengaruhi strategi perawatan pembedahan. Oleh itu, artikel ini cuba melihat faktor-faktor yang mempengaruhi perawatan patah tulang metatarsal kelima.', 1),
(29551, 29541, 'Skizofrenia merupakan penyakit mental yang kronik dan dicirikan dengan gangguan persepsi, pemirikiran, kognitif dan perubahan tingkah-laku. Salah satu daripada gejala utama skizofrenia ialah halusinasi auditori. Di dalam laporan ini, kita membincangkan kes seorang lelaki pelarian Rohigya yang berumur  31 tahun yang telah mengamputasikan zakar beliau secara total akibat dari halusinasi arahan yang diterima oleh beliau, di mana beliau telah pun sakit sejak sekian lama di Myanmar. Beliau telah dikejarkan ke hospital UKM untuk mendapatkan rawatan. Setelah keadaan beliau stabil, beliau telah dimasukkan ke wad psikiatri untuk pemerhatian dan rawatan perubatan selanjutnya. Beberapa isu berkaitan dengan factor psikososial,  ekonomi dan politik menjadikan kes ini unik untuk dibincangkan. Penama telah dirawat dengan ubat sulpride dan telah didiscaj ke pihak pembedahan urologi untuk rawatan selanjutnya.', 1),
(25510, 25501, '<p>\r\n	Dua petanda kesihatan salur darah baru yang dihasilkan daripada gelombang fotopletismografi jari (PPG) telah diperkenalkan berdasarkan populasi rakyat Malaysia iaitu indeks kecergasan PPG (PPGF) dan indeks jangkaan risiko salur darah (VRPI). Antara objektif kajian ini adalah untuk mengkaji hubungan antara PPGF dengan petanda penyakit jantung (CVD) yang lain seperti halaju gelombang denyutan karotid femoral (PWVCF), untuk membandingkan PPGF antara mereka yang sihat dan mempunyai faktor risiko CVD dan untuk menentukan sensitiviti VRPI dalam mengenalpasti mereka yang mempunyai faktor risiko CVD. Seramai 114 lelaki berumur antara 20 hingga 40 tahun yang sihat atau mempunyai mempunyai faktor risiko CVD telah direkrut. Faktor risiko CVD termasuk hipertensi, merokok, dislipidemia, obesiti abdomen dan sejarah keluarga berpenyakit CVD pramatang. Subjek dibahagikan kepada kumpulan sihat, mereka yang mempunyai satu faktor risiko dan mereka yang mempunyai dua atau lebih faktor risiko. Berat, tinggi, tekanan darah (BP) periferi dan pusat, PWVCF dan PPGF mereka diukur dan sensitiviti VRPI dalam mengenal pasti mereka yang berisiko CVD dikira. Data dianalisis dengan menggunakan SPSS versi 15 dan nilai P&lt;0.05 adalah signifikan. Purata umur subjek adalah 28.94&plusmn;4.86 tahun. Tiada perbezaan pada PPGF didapati antara kumpulan-kumpulan (P&gt;0.05). Pembolehubah tidak bersandar untuk PPGF adalah tekanan hadapan (Beta=0.35,P&lt;0.01), PWVCF (Beta=-0.26,P&lt;0.01), BP sistolik (Beta=-0.26,P=0.04) dan ketinggian (Beta=0.24,P&lt;0.01). Sensitiviti VRPI adalah 82.02%. Kesimpulannya, PPGF adalah berkait dengan PWVCF dan berpotensi untuk menjadi petanda kekenyalan arteri. Tambahan lagi, VRPI adalah sensitif untuk digunakan sebagai petanda untuk saringan awal faktor risiko CVD.</p>\r\n', 1),
(25518, 7244, 'Pembentukan semula tulang diatur oleh keseimbangan yang berterusan antara penyerapan dan pembentukan. Gangguan keseimbangan tersebut mungkin membawa kepada gangguan tulang metabolik seperti osteoporosis. Pengukuran histomorfometri tulang diperlukan untuk memahami kesan penyakit tulang dan juga keberkesanan rawatan pada metabolisme tulang dalam satu tempoh masa. Kajian sebelum ini menggunakan teknik Weibel sebagai kaedah stereologikal kuantitatif untuk menentukan perubahan sel dan dinamik tulang. Walau bagaimanapun, tidak ada penerangan tentang bagaimana teknik ini telah digunakan. Kajian ini bertujuan untuk memberikan gambaran lengkap tentang penggunaan teknik Weibel untuk mengukur indeks histomorfometri tulang statik dan dinamik. Teknik Weibel menyediakan maklumat yang boleh dipercayai mengenai metabolisme tulang. Walau bagaimanapun, kepakaran teknikal, pemprosesan optimum sampel tulang, perawakan hirisan tisu trabekular dan jumlah imej teranalisa yang mencukupi untuk setiap bahagian adalah diperlukan untuk mencapai keputusan yang boleh dipercayai dengan kemungkinan ralat yang rendah.', 1),
(29584, 29574, '<p>\r\n	Penggunaan Perubatan Komplementari dan Alternatif (PKA) dalam kalangan pesakit yang mengidap penyakit kronik adalah semakin popular. Kajian ini merupakan kajian deskriptif keratan rentas yang dilakukan di 45 klinik kesihatan kerajaan di Negeri Sembilan untuk menentukan prevalen penggunaan PKA. Responden di klinik diabetes dipilih melalui pensampelan rawak bersistematik dan ditemu ramah menggunakan borang soal selidik yang berstruktur. Penggunaan PKA dibahagikan kepada tiga kumpulan: PKA untuk diabetes (PKA-DM), PKA untuk kesihatan umum (PKA-K) dan Bukan Pengguna. Prevalen penggunaan PKA dalam kalangan pesakit diabetes jenis II di Negeri Sembilan adalah 58.5% (PKA-DM: 40.6% dan PKA-K: 17.9%). Bagi kumpulan PKA-DM, peria (Momordica charantia) merupakan PKA paling popular untuk membantu kawalan diabetes, manakala susu suplemen merupakan pilihan paling popular untuk kumpulan PKA-K. Kesimpulannya, penggunaan PKA dalam kalangan pesakit diabetes jenis II di Negeri Sembilan adalah lazim. Produk semulajadi adalah pilihan utama jenis PKA untuk membantu kawalan diabetes. Kebanyakan pengguna PKA tidak memberitahu anggota kesihatan tentang penggunaan PKA mereka.</p>\r\n', 1),
(29692, 29682, '<p>\r\n	Objektif: Untuk membandingkan insiden tekanan dalam mata yang tinggi di kalangan pesakit tidur berapnea dan pesakit tanpa tidur berapnea. Jenis kajian: Ini adalah sebuah kajian rentas selama satu tahun Tempat: Kajian ini dijalankan di Jabatan Otorinolaringologi, UKMMC, sebuah hospital rujukan tertiari. Subjek: 50 subjek dengan masalah tidur berapnea (OSA) dan 50 subjek tanpa masalah tidur berapnea (non OSA) telah masuk ke dalam kajian ini. Purata umur adalah 37.0 tahun (19,65) dalam populasi etnik bercampur (76% (Melayu), 18% (Cina) and 6% (India)) Keputusan: Kesemua pesakit telah melaui ujian tidur menggunakan mesin polisomnogram (SOMNOCheck Effort Weinmann, Hamburg, Germany), Epworth Sleepiness Scale (ESS), Mullers menuevre (MM) untuk menguji peratus penyumbatan dan tonometri. Insiden tekanan dalam mata yang tinggi di kalangan subjek dengan tidur berapnea (OSA) adalah 52% dengan perbezaan signifikan di antara subjek non-OSA and OSA . Korelasi antara ESS and IOP adalah signifikan (p&lt;0.05) untuk kedua belah mata ( right eye r&sup2; = 0.24, left eye r&sup2; = 0.28). Terdapat perbezaan signifikan antara purata IOP antara subjek non-OSA dan OSA untuk kedua belah mata (p&lt; 0.05). Perhubungan yang signifikan telah dijumpai antara penyumbatan di bahagian retropalatal dan retrolingual dengan IOP tinggi. (p&lt;0.05) Kesimpulan: Dapatan kajian menunjukkan insiden IOP tinggi di kalangan subjek dengan OSA. Terdapat perhubungan signifikan antara parameter OSA (ESS, penyumbatan dalam MM) dengan IOP tinggi. Kami mengesyorkan pemeriksaan periodik tahap IOP di kalangan pesakit OSA.</p>\r\n', 1),
(29697, 29687, '<p>\r\n	Abstrak Trakeostomi boleh dikaitkan dengan pelbagai komplikasi. Disini, kami menulis kes berkaitan kecuaian perubatan terhadap pesakit dimana seorang pesakit telah menggunakan tiub trakeostomi melebihi jangkamasa tanpa menukarnya menyebabkan komplikasi tisu parut kulit yang besar pada trakeostoma dan tisu parut di dalam trakea yang menyebabkan kedudukan tiub trakeostomi teralih dari kedudukan asal seterusnya mengakibatkan halangan pada salur pernafasan. Kes ini menggambarkan bahawa kesedaran dan pengetahuan berkaitan penjagaan tiub trakeostomi yang betul di kalangan kakitangan perubatan adalah sangat penting bagi mengelakkan sebarang salah urus yang boleh mengakibatkan komplikasi yang mengancam nyawa kepada pesakit.</p>\r\n', 1),
(29702, 29692, '<p>\r\n	Selulitis orbital merupakan jangkitan tisu di sekitar mata yang melibatkan tisu-tisu lemak dan otot yang melepasi septum orbital. Ia boleh menyebabkan komplikasi yang mengancam penglihatan pesakit serta boleh membawa kematian jika tidak dirawat sebaiknya. Diagnosis serta rawatan awal amat penting untuk mengelakkan sebarang komplikasi buruk. Penyebab jangkitan yang lazim adalah disebabkan oleh jangkitan di bahagian sinus yang merebak melalui lapisan nipis lamina papyracea atau melalui jangkitan pan-oftalmitis. Kes ini memberi penerangan terperinci mengenai jangkitan selulitis orbital yang di luar kebiasaan melalui luka torehan pada konjunktiva mata.</p>\r\n', 1),
(29760, 29750, 'Kongenital Hidung Pyriform Aperture Stenosis (CNPAS) adalah penyakit yang amat jarang menyebabkan kesempitan saluran udara hidung. Secara klinikal, ia seperti atresia choanal pada bayi. Ia perlu dibezakan kerana pengurusan kedua-dua penyakit ini adalah berbeza. Bukaan pyriform hidung adalah bahagian yang paling hadapan dan sempit saluran udara hidung. Walaupun sedikit kesempitan di kawasan keratan rentas hidung, ia akan meningkatkan rintangan saluran udara hidung. CNPAS biasanya dikaitkan dengan kecacatan pembangunan garis tengah seperti kekurangan holoprosensefali atau pituitari. Kami menemui kes yang pertama dilaporkan CNPAS yang berkaitan denganTrisomi 8 Mosaicsm (T8M).', 1),
(29804, 29794, '<p>\r\n	Tomografi berkomputer mikro (Micro-CT) adalah peralatan yang berguna untuk memantau pemulihan patah tulang pada model osteoporosis. Ia menyediakan imej 3D kalus fraktur yang boleh digunakan untuk mengira parameter tulang secara kuantitatif. Dalam kajian in, micro-CT telah digunakan untuk menilai pemulihan patah tulang tikus yang diorkidektomi, iaitu model osteoporosis defisien-androgen. Kesan ke atas pemulihan patah tulang oleh Eurycoma longifolia, sejenis tumbuhan ubatan dengan aktiviti pro-androgenik, dinilai menggunakan micro-CT. Tikus dibahagikan kepada beberapa kumpulan : kawalan orkidektomi (ORX), bedah sham (SHAM), orkidektomi dan disuntik dengan testosteron secara intramuskular seminggu sekali (TEN) dan orkidektomi dan menerima oral gavaj Eurycoma longifolia (EL) setiap hari. Jangkamasa rawatan ialah enam minggu. Kepatahan diaruh ke atas tibia kanan kesemua tikus. Mikro-CT telah digunakan untuk mengimbas kalus fraktur yang terdiri daripada 100 potongan aksial di atas dan di bawah aras fraktur. Analisa micro-CT berjaya mengesan perbezaan signifikan pada pemulihan fraktur kumpulan ORX dan TEN. Bagaimanapun, tiada perubahan micro-CT yang signifikan dilihat pada pemulihan fraktur kumpulan EL. Kesan EL ke atas pemulihan fraktur tidak dapat dilihat pada model tikus orkidektomi.</p>\r\n', 1),
(29805, 29795, 'Seorang wanita berusia 58 tahun yang menghidap penyakit epilepsi telah datang ke jabatan kecemasan  kerana mengadu sakit badan setelah mendapat serangan sawan tonik-klonik yang berlaku beberapa jam sebelumnya. Semasa pemeriksaan, beliau didapati berada dalam kesakitan serta tidak berupaya untuk menggerakkan kesemua anggota badannya. Selepas diberikan ubat tahan sakit, beliau masih mengadu  berasa sakit serta tidak boleh menggerakkan kedua-dua bahunya. Pemeriksaan lanjutan menunjukkan pergerakan yang terhad dalam semua arah pada kedua-dua bahu beliau namun tiada abnormaliti kelihatan pada struktur bahunya. X-ray pada kedua-dua bahagian bahu beliau menunjukkan kepatahan pada bahagian proksimal tulang humerus kiri dan kanan. Kepatahan bahagian proksimal tulang humerus jarang berlaku dengan sendirinya berikutan sawan; mereka selalu dikaitkan dengan dislokasi sendi glenohumeral. Di sini, kami melaporkan satu kes yang mana kami hampir terlepas mendiagnosa kepatahan di bahagian proksimal humerus selepas serangan serangan sawan tonik-klonik.', 1),
(29864, 29854, 'Penilaian risiko penyakit kardiovaskular (CVD) adalah amat penting dalam pengesanan awal CVD pada pesakit asimptomatik. Fotopletismogram (PPG) jari adalah teknik bukan invasif digunakan yang mengesan perubahan isipadu darah dalam mikrovaskular tisu jari. Indeks kecergasan PPG (PPGF) yang didapati dari satu perisian yang dibina di Universiti Kebangsaan Malaysia telah menunjukkan dapat meramal umur vaskular. Kajian ini bertujuan untuk menentukan keupayaan PPGF dalam mengklasifikasikan subjek dari segi statu CVD subjek. Di samping itu, sensitiviti dan spesifisiti PPGF dalam membezakan subjek sihat dari subjek dengan faktor-faktor risiko kardiovaskular konvensional dan penyakit arteri koronari (CAD) ditentukan. Dalam kajian ini, PPGF telah dirakam menggunakan NivariX dari tiga kumpulan yang berbeza (n = 30 bagi setiap kumpulan, berumur: 45 - 55 tahun), pesakit, berisiko tinggi dan sihat. Kumpulan pesakit merujuk kepada pesakit CAD yang didiagnosis dari angiogram dan kumpulan berisiko tinggi merujuk kepada yang didiagnosis mempunyai lebih daripada dua faktor risiko kardiovaskular (sejarah keluarga, diabetes, hipertensi, hiperlipidemia atau perokok). Analisis Kovarians (ANCOVA) dengan usia dan tekanan darah sebagai faktor pembaur telah menekankan PPGF dalam tiga kumpulan yang berbeza. PPGF dalam kumpulan sihat adalah paling tinggi pada 59.94 ± 7.80%, berbanding dengan subjek yang berisiko tinggi: 48.84 ± 8.14% dan pesakit: 52.61 ± 9.90% (p <0.01). Analisis Fungsi Diskriminan telah dilakukan untuk menilai ketepatan PPGF dalam mengklasifikasikan subjek sebagai pesakit, kumpulan berisiko tinggi atau sihat. Hasil analisis menunjukkan bahawa PPGF mempunyai sensitiviti 60.0% dan spesifisiti 63.3%. Spesifisiti yang agak baik bagi peranti PPG ini menunjukkan ia mempunyai potensi sebagai alat saringan CVD di pusat-pusat penjagaan primer.', 1),
(29964, 29954, '<p>\r\n	Cytomegalovirus (CMV) Retinitis merupakan penyakit yang berlaku pada pesakit yang menghidapi Human Immunodeficiency Virus (HIV) terutamanya. Ia juga dilaporkan pada pesakit tanpa HIV, contohnya pesakit menghidapi gangguan autoimun yang memerlukan immunosuppresif, pesakit menjalani pemindahan organ atau sumsum tulang, pesakit kanser darah atau payudara yang menerima kemoterapi, penyakit mata berikutan suntikan kortikosteroid intraokular atau periokular, kencing manis dan Good sindrom. Walau bagaimanapun, kes CMV retinitis dalam pesakit yang menghidapi dermatomyositis dan kanser pada masa yang sama tidak pernah dilaporkan sebelum ini. Ia juga tidak dilaporkan dalam kanser selain kanser darah atau payudara, atau pesakit kanser sebelum menerima rawatan kemoterapi. Kami melaporkan kes CMV retinitis dalam seorang wanita tanpa HIV berusia 40 tahun yang menghidapi dermatomyositis dan kanser paru-paru, di mana CMV retinitisnya kembali apabila immunosuppresif dimula semula. Kedua-dua episod CMV retinitis telah berjaya dirawat selepas mengambil kira keadaan keimunannya.</p>\r\n', 1),
(29969, 29959, '<p>\r\n	Abstrak Kongenital Hidung Pyriform Aperture Stenosis (CNPAS) adalah penyakit yang amat jarang menyebabkan kesempitan saluran udara hidung. Secara klinikal, ia seperti atresia choanal pada bayi. Ia perlu dibezakan kerana pengurusan kedua-dua penyakit ini adalah berbeza. Bukaan pyriform hidung adalah bahagian yang paling hadapan dan sempit saluran udara hidung. Walaupun sedikit kesempitan di kawasan keratan rentas hidung, ia akan meningkatkan rintangan saluran udara hidung. CNPAS biasanya dikaitkan dengan kecacatan pembangunan garis tengah seperti kekurangan holoprosensefali atau pituitari. Kami menemui kes yang pertama dilaporkan CNPAS yang berkaitan denganTrisomi 8 Mosaicsm (T8M).</p>\r\n', 1),
(31577, 31567, 'Penyembuhan luka merupakan proses  kompleks yang melibatkan homeostasis, keradangan, proliferasi, dan kematangan serta pembentukan semula. Keempat-empat fasa berlaku dalam urutan dan tempoh masa yang betul. Kebanyakan luka sembuh tanpa kesukaran. Walau bagaimanapun, sesetengah luka tidak akan sembuh dengan betul kerana kehadiran faktor-faktor yang menjejaskan penyembuhan luka. Luka yang tidak sembuh boleh menjadi kronik. Dalam keadaan kencing manis, penyembuhan luka mungkin terjejas. Individu diabetes cenderung untuk menghidap ulser kaki diabetik (DFU). Spesies oksigen reaktif (ROS) memainkan peranan yang penting dalam diabetes. Individu diabetes mempunyai tahap antioksidan yang rendah. Vitamin E menunjukkan bahawa ia wujud sebagai antioksidan. Selain daripada kesan sistemik, vitamin E memberikan kesan topikal. Ianya telah ditetapkan penggunaannya terutama dalam produk dermatologi. Namun, keputusannya tetap bercanggah. Walau bagaimanapun, kesan vitamin E pada penyembuhan luka masih kontroversi. Banyak kajian telah dijalankan untuk melihat keupayaan vitamin E, terutamanya dalam modulasi transduksi isyarat dan ekspresi gen. Walau bagaimanapun, mekanisme molekul yang terperinci masih belum dapat dijelaskan. Vitamin E boleh mengawal penghasilan ROS melalui kesan perencatan vitamin E terhadap aktiviti dan ungkapan enzim penghasilan ROS. Kesan vitamin E boleh bergantung kepada kepekatan dan kehadiran oksidan / antioksidan. Objektif artikel ini ialah untuk mengkaji semula peranan dan mekanisme tindakan vitamin E dalam proses penyembuhan luka sama ada di peringkat selular atau molekul.', 1),
(32699, 32689, '<p>\r\n	Keresahan and gejala mood amat biasa kedapatan di dalam Penyakit Kecelaruan Defisit Perhatian. Laporan kes ini menengahkan kes memperincikan 23 tahun lelaki Melayu telah menunjukkan masalah memberi perhatian dan kemerosotan pencapaian akademiknya secara ketara. Kemerosotan pengajiannya disedari oleh ahli keluarganya sebelum 2-3 years sebelum beliau mendapatkan rawatan psikiatri. Beliau juga mengalamai masalah mood rendah dan walaubagaimana pun tidak mengalami gejala biologi kemurungan. Beiau telah dirawat dengan ubat-ubatan Buproprion 150mg setiap hari dan juga ubat Ritalin 10mg. Beliau telah menunjukkan penambah-baikan seleoas memakan ubat dan seterusnya dapat member fokus kepada pembelajaran akademiknya.</p>\r\n', 1),
(38204, 38194, '<p>\r\n	Objektif: Untuk menyelidik impak ubat suntikan mata iaitu Ranibizumab kepada kualiti hidup pesakit yang menghadapi penyakit &lsquo;neovascular age-related macular degeneration&rsquo; (AMD). Methodologi: Prospektif kes bersiri, melibatkan 110 pesakit yang belum pernah mendapat rawatan, dan selepas 3 kali suntikan ubat 0.5mg Ranibizumab. Selain pemeriksaan mata yang menyeluruh, kualiti hidup diselidik dengan menggunakan borang soal selidik NEI-VFQ-25. Keputusan: Purata umur pesakit ialah 66 tahun (45-83 tahun), 62 pesakit (56%) ialah lelaki, 58 pesakit (53%) ialah kaum Cina, 35 pesakit (32%) ialah Melayu dan 17 pesakit (15%) kaum India. Kebanyakan mata yang terlibat ialah mata yang lebih teruk kadar penglihatan, n=95 (86%), dan 71 pesakit hanya satu mata sahaja menghadapi penyakit ini. Purata penglihatan mata pesakit meningkat dari 0.8 (logMAR) sebelum rawatan, kepada 0.4 tahap penglihatan selepas rawatan. Skor fungsi kualiti hidup meningkat dari purata markah 53 kepada 80, purata perbezaan ialah 27 markah ((p= 0.000, &lt;0.05). Selepas 3 bulan, purata perbezaan dalam skor kualiti hidup semua kumpulan pesakit meningkat dalam semua subskala, dan subskala yang paling tinggi ialah kumpulan fungsi memandu dan aktiviti deka. Peningkatan skor adalah ketara mengikut statistik (p=0.000, &lt;0.05). Kesimpulan: Ubat suntikan mata Ranibizumab ialah suatu rawatan yang sangat berharga dalam merawat penyakit &lsquo;neovascular AMD&rsquo;. Selain daripada peningkatan yang ketara dalam tahap penglihatan mata, ubat ini juga meningkatkan tahap kualiti kehidupan pesakit.</p>\r\n', 1),
(38219, 38209, '<p>\r\n	Euphorbia tirucalli dilaporkan mempunyai aktiviti antibakteria terhadap pelbagai mikroorganisma. Kajian in vitro ini bertujuan untuk menilai ciri-ciri antibakteria extrak (metanol, etanol dan ekstrak akueus) batang Euphorbia tirucalli terhadap bakteria yang berkaitan dengan karies gigi, iaitu Streptococcus mutans dan Streptococcus sobrinus. Sifat-sifat antibakteria telah ditentukan menggunakan ujian resapan agar berlubang pada kepekatan ekstrak yang berbeza (10, 20 dan 30 mg / ml). Komersial amoxicillin (10 &mu;g) telah digunakan sebagai kawalan positif manakala pelarut yang sesuai telah digunakan sebagai kawalan negatif. Ekstrak metanol dan ethanol daripada batang Euphorbia tirucalli didapati berkesan terhadap S. mutans dan S. sobrinus. Walau bagaimanapun, ekstrak akueus batang Euphorbia tirucalli tidak menunjukkan aktiviti terhadap kedua-dua strain bakteria. Perbezaan dalam ciri-ciri antibakteria dalam perbezaan ekstrak Euphorbia tirucalli mungkin disebabkan oleh perbezaan dalam juzuk fitokimia.</p>\r\n', 1),
(41571, 41561, '<p>\r\n	Helicobacter pylori merupakan penyebab utama gastritis kronik dan penyakit ulser peptik. Jangkitan kronik H. pylori dikaitkan dengan peningkatan risiko mendapat barah perut. H. pylori telah diketahui mempunyai kepelbagaian genetik dengan hampir setiap individu dijangkiti oleh populasi bakteria yang berbeza. Variasi genotip H. pylori mungkin mempunyai kaitan dengan perbezaan faktor-faktor virulens dalam kalangan strain dan ini ditunjukkan melalui hasil klinikal berbeza pada pesakit yang dijangkiti. Artikel ulasan ini meringkaskan kepelbagaian genetik populasi H. pylori, faktor pelekatan dan virulens H. pylori yang berhubungkait dengan berbagai penyakit akibat jangkitan H. pylori dalam populasi manusia di kawasan geografi yang berbeza.</p>\r\n', 1),
(42049, 42039, '<p>\r\n	Epidermal naevus adalah hamartoma kulit kongenital yang benigna. Kami mengetengahkan kes yang jarang ditemui iaitu epidermis naevus dengan transformasi malignan untuk karsinoma sel basal. Seorang lelaki Cina berusia 79 tahun telah dibiopsi untuk nodul kulit berukuran 2 x 4 cm yang timbul di dalam lesi papular linear berukuran 2 x 4 cm di sebelah kiri lehernya. Nodul itu kemudiannya disahkan sebagai karsinoma sel basal yang timbul daripada epidermal naevus. Mutasi gen PIK3CA mutasi dikaitkan dengan karsinoma sel basal, yang menunjukkan komponen sel karsinoma basal adalah bebas daripada komponen epidermis naevus. Doktor klinikal dan pakar patologi perlu sedar kemungkinan perubahan malignan yang mungkin timbul dalam epidermal naevus.</p>\r\n', 1),
(42879, 42869, '<p>\r\n	Objektif: Autisme (ASD) merupakan satu jenis ketidakseimbangan perkembangan neuro kanak-kanak yang dikaitkan dengan kecacatan kognitif dan bahasa. Penyelidikan sebelum ini mendapati bahawa kanak-kanak yang mempunyai ketidakseimbangan perkembangan meningkatkan tahap tekanan ibu bapa. Namun, ibu bapa yang mempunyai anak ASD mengalami tahap tekanan yang lebih tinggi berbanding ibu bapa yang mempunyai kanak-kanak ketidakseimbangan perkembangan yang lain. Justeru, kajian ini bertujuan untuk mengkaji perbezaan tahap tekanan antara ibu bapa yang mempunyai kanak-kanak ASD dan ibu bapa yang mempunyai kanak-kanak perkembangan tipikal (TD) yang dikategorikan dalam kumpulan kontrol. Methodologi: Parenting Stress Index, terbitan ketiga digunakan untuk mengakses tahap tekanan dalam kalangan 30 ibu bapa yang mempunyai kanak-kanak ASD dan 36 ibu bapa yang mempunyai kanak-kanak TD. Sampel ibu bapa yang mempunyai kanak-kanak autisme dikumpul dari Pusat Perubatan Universiti Kebangsaan Malaysia, Hospital Tangkak, dan Pusat Autisme (NASOM) di Muar dan Segamat. Hasil Penyelidikan: Ibu bapa yang mempunyai kanak-kanak ASD mempunyai kadar tekanan yang signifikanlebih tinggi berbanding ibu bapa mempunyai kanak-kanak TD (p&lt;0.001). Ibu bapa yang mempunyai kanak-kanak lelaki ASD mempunyai kadar tekanan yang signifikan lebih tinggi berbanding ibu bapa yang mempunyai kanak-kanak lelaki TD (p&lt;0.001). Ibu bapa yang mempunyai kanak-kanak perempuan ASD mempunyai kadar tekanan yang signifikan lebih tinggi berbanding ibu bapa yang mempunyai kanak-kanak perempuan TD (p&lt;0.001). Ibu mencatat tahap stress yang lebih tinggi berbanding ayah namun tiada perbezaan yang signifikan dari segi tahap stress antara ibu dan ayah yang mempunyai kanak-kanak ASD. Ibu bapa yang mempunyai kanak-kanak ASD yang berumur lebih besar menunjukkan tahap tekanan yang lebih tinggi tetapi tiada perbezaan signifikan antara ibu bapa yang mempunyai kanak-kanak ASD dalam lingkungan umur yang berbeza.</p>\r\n', 1),
(46094, 46084, '<p>\r\n	Fusarium adalah salah satu daripada patogen yang paling penting dalam bijirin; jangkitan kulat telah meningkat pada tahun-tahun kebelakangan ini kerana perubahan ekologi dan peningkatan kandungan nitrogen tanah. Dalam kajian semasa kesan intipati minyak jintan putih pada verticillioides Fusarium dengan kaedah molekul disiasat. Kajian ini melibatkan 14 pencilan, 11 pencilan verticillioides Fusarium dan 3 Fusarium bukan verticillioides sampel (kawalan negatif) telah diuji. Untuk ujian MIC dan MFC, budaya RPMI1640 dalam microplate telah digunakan. Selepas seminggu, sampel RNA berbudaya menggunakan kit pengekstrakan RNA daripada Fermentas syarikat (Purification Mini Kit GeneJET Plant RNA) diekstrak daripada biojisim kulat dan ujian RT-PCR bersama-sama dengan gen FUM1 yang dilakukan. Analisis RT-PCR dipaparkan bahawa FUM1 gen dinyatakan dalam sampel kulat yang bukan di bawah kesan intipati minyak, tetapi gen tidak dinyatakan dalam sampel selepas kesan jintan putih dan tidak ada mana-mana band di gel agarose. toksin B1 fumonisin yang dihasilkan oleh kulat Fusarium verticillioides mengandungi beberapa gen. The FUM1was gen terlibat toksin gen sintesis laluan toksin ini sebagai gen yang paling penting.</p>\r\n', 1),
(46452, 46442, '<p>\r\n	LATAR BELAKANG: Penggantian cecair adalah rawatan utama untuk kencing manis ketoacidosis (DKA). Pada masa ini pilihan terbaik daripada cecair masih diperdebatkan. 0.9% Normal Saline (NS) yang menyebabkan ketidakseimbangan metabolik dan asidosis metabolik biasanya digunakan. Sterofundin&reg; merupakan kristaloid alternatif yang dianggap mempercepatkan resolusi asidosis. Kelebihan dalam kandungan sterofundin adalah perbezaan kecil besar ion (SID) kepada plasma dan kandungan klorida yang lebih rendah. OBJEKTIF: Objektif utama adalah untuk membandingkan kadar resolusi asidosis dalam pesakit DKA antara rawatan dengan 0.9% Saline Normal dan Sterofundin&reg; dalam 12 jam. Objektif lain adalah untuk membandingkan perbezaan yang signifikan ion (SID), 12 jam pelepasan keton darah dan keseimbangan elektrolit antara kedua-dua kumpulan. KAEDAH: Kajian ini adalah kajian terbuka dilabel percubaan trial kawalan rawak. Kajian ini dijalankan selama 6 bulan. KEPUTUSAN: Saiz sampel sebanyak 18 orang telah diperolehi dan (n = 9) untuk setiap lengan. Perbezaan utama antara kedua-dua kumpulan adalah awal median 2 jam peningkatan tahap pH (NS = 0,006 vs Sterofundin = 0,05, P = 0,063), bagaimanapun tidak ketara. Keton, pengurangan jurang anion, normalisasi bikarbonat, sodium, klorida, urea dan tahap kreatinin gagal menunjukkan sebarang perbezaan yang signifikan antara kedua-dua kumpulan. Dua belas jam paras klorida median kenaikan adalah lebih tinggi dalam kumpulan NS (11) berbanding dengan kumpulan sterofundin yang (6). Tidak ada perbezaan antara kematian dan morbiditi. KESIMPULAN: Perbandingan kedua-dua kumpulan cecair, tidak terdapat perbezaan biokimia penting sepanjang rawatan DKA. Walau bagaimanapun saiz sampel yang kecil dan memerlukan pengesahan selanjutnya daripada ujian yang lebih besar.</p>\r\n', 1),
(49280, 49270, '<p>\r\n	Penyakit kanser sel basal (KSB) di bahagian skrotum jarang ditemui dan punca ia berlaku tidak difahami dengan baik. KSB yang tumbuh di skrotum ini dikatakan lebih agresif dan mempunyai kebolehan merebak yang lebih tinggi daripada KSB kawasan-kawasan lain. Rawatan utama untuk KSB peringkat awal di skrotum adalah pembedahan, manakala untuk peringkat lanjut di mana penyakit telah merebak, ia perlu dikawal dengan rawatan kemoterapi. Satu keadaan klinikal yang luarbiasa di mana KSB skrotum berlaku serentak dengan kanser yang lain tidak pernah dilaporkan. Kes ini mengisahkan cabaran diagnostik dan rawatan kerana KSB skrotum dan kanser sel squamous (KSS) berlaku pada waktu yang sama. Seorang lelaki berusia 70 tahun mengadu wujudnya ulcer di skrotum beliau yang tidak sembuh selama lebih setahun. Pada ketika itu, beliau sedang disiasat kerana mengalami penyakit KSS di telinga yang berada di peringkat lanjut. Ulcer di skrotum itu pada mulanya dianggap sebagai KSS yang telah merebak ke situ. Ia kemudiannya didapati penyakit KSB dengan biopsi dan pemeriksaan histopatologi. Beliau menjalani pembedahan untuk membuang ulcer di skrotum dan diikuti dengan kemoterapi paliatif menggunakan cisplatin dan radioterapi ke atas kumpulan-kumpulan limpa yang dijangkiti KSS. Enam bulan selepas tamatnya rawatan paliatif, beliau kekal sihat dan tidak mempunyai apa-apa kesan KSB berulang di skrotum. Rawatan pembedahan bersama dengan kemoterapi dan radioterapi paliatif dapat memberi kualiti hidup yang baik untuk pesakit yang ditimpa KSB skrotum dan KSS peringkat lanjut.</p>\r\n', 1),
(50713, 50703, '<p>\r\n	Abstrak Latarbelakang: Dos Subhypnotic propofol memberikan kesan anaelgesia yang effektif dan berpanjangan selepas kelahiran Caesarean tetapi menyebabkan tidak keselesaan kerana kegatalan yang disebabkan oleh spina morfin. Objektif: Kajian prospektif, rabun dua hala ini membandingkan dos subhipnotik propofol sebagai profilaksis untuk kegatalan yang disebabkan oleh spina morfin. Bahan dan tatacara: Seramai 53 ibu mengandung, ASA I &amp; II yang menjalani kelahiran Caesarean secara elektif dibahagikan kepada kumpulan propofol 20 mg (Kumpulan A), propofol 10 mg (Kumpulan B) and &lsquo;normal saline&rsquo; (Kumpulan C) secara rambang. Semua ibu mengandung menerima 0.5% bupivacaine hiperbarik 2 ml, fentanyl 25 &mu;g dan morfin 0.15 mg sebagai pembiusan spina. Selepas kelahiran bayi, ubat kajian diberikan sebagai bolus perlahan dalam 30 saat. Kegatalan yang disebabkan oleh spina morfin dinilai menggunakan &lsquo;Verbal Rating Scale&rsquo; and direkodkan semasa di ruang pemulihan, 4, 8 and 12 jam selepas pemberian spina morfin. Keputusan: Data demografik pesakit ketiga-tiga kumpulan adalah setara. Kejadian kegatalan secara terkumpul adalah 29.4% (95% CI: 7,8-51,1%) dalam Kumpulan A dan B, dan 52.7% (95% CI: 30,2-75,1%) dalam Kumpulan C tetapi tidak ketara secara statistik, p = 0.25. NNT untuk kegatalan sederhana dan teruk adalah 3.23%(95% CI: 2.99-3.47) dalam Kumpulan A dan 3.98; 95%(CI: 3.71-4.25) dalam Kumpulan B. Insiden kegatalan yang disebabkan oleh spina morfin yang sederhana adalah lebih ketara rendah di dalam Kumpulan A berbanding dengan Kumpulan C hanya pada jam ke- 4 (p &lt; 0.05). Kesimpulan: Kajian ini menunjukkan dos subhipnotik propofol adalah tidak effektif dalam mengurangkan kegatalan yang disebabkan oleh spina morfin yang disebabkan oleh spina morfin semasa kelahiran Caesarean. Walau bagaimanapun, kadar kegatalan berkurangan dengan 20mg propofol pada jam ke-4 selepas spina morfin.</p>\r\n', 1),
(50782, 27866, '<p>\r\nSolar retinopathi merupakan satu penyakit retina yang disebabkan oleh pandangan terhadap cahaya matahari. Diagnosis penyakit ini amat mencabar dan memerlukan sejarah kesihatan dan penyiasatan yang teliti. Laporan kes ini adalah berkenaan seorang pemetik buah-buahan yang berumur 55 tahun yang mengalami kehilangan penglihatan kekal di bahagian tengah medan penglihatan. Diagnosis solar retinopathi telah dibuat berdasarkan kepada sejarah yang diberi oleh pesakit berkenaan, bersama penyiasatan menggunakan pengimejan seperti optical coherence tomography (OCT), infrared (IF) dan fundus autofluorescence (FAF) yang memainkan peranan penting dalam kes ini. Kesimpulannya, solar retinopathi adalah satu penyakit yang sukar untuk dipulihkan. Penyiasatan dengan cara pengimejan memainkan peranan yang penting untuk merangkul diagnosis yang betul. Justeru itu, pemakaian cermin mata hitam adalah disarankan untuk semua pesakit yang sering terdedah kepada cahaya matahari. \r\n', 1),
(51616, 29687, '<p>\r\n	Abstrak Trakeostomi boleh dikaitkan dengan pelbagai komplikasi. Disini, kami menulis kes berkaitan kecuaian perubatan terhadap pesakit dimana seorang pesakit telah menggunakan tiub trakeostomi melebihi jangkamasa tanpa menukarnya menyebabkan komplikasi tisu parut kulit yang besar pada trakeostoma dan tisu parut di dalam trakea yang menyebabkan kedudukan tiub trakeostomi teralih dari kedudukan asal seterusnya mengakibatkan halangan pada salur pernafasan. Kes ini menggambarkan bahawa kesedaran dan pengetahuan berkaitan penjagaan tiub trakeostomi yang betul di kalangan kakitangan perubatan adalah sangat penting bagi mengelakkan sebarang salah urus yang boleh mengakibatkan komplikasi yang mengancam nyawa kepada pesakit.</p>\r\n', 1),
(51992, 51980, '<p>\r\n	Intravaskular papillary endothelial hyperplasia (IPEH) atau Masson&rsquo;s tumor adalah sejenis benign vaskular percambahan yang meniru malignan vaskular tumor yang lain. Sehingga kini, kes ini merupakan kes Masson&rsquo;s tumor pada uretra yang keempat yang pernah dilaporkan dalam kajian atau laporan yang sedia ada. Seorang wanita yang berusia 65 dan para 2 dengan masalah pendarahan selepas menopause. Pemeriksaan menunjukkan satu ketumbuhan bersaiz 4x3x3 cm sekitar uretra meatus. Pesakit telah menjalani pemeriksaan di bawah bius dan pemotongan ketumbuhan sekitar uretra. Pemeriksaan histopatologi mengesahkan Masson&rsquo;s tumor. Diagnosis dan pengendalian kes luar biasa ini di lokasi yang jarang berlaku dibincang dalam kes laporan ini.</p>\r\n', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(52835, 52823, '<p>\r\n	Kaedah penyimpanan mayat manusia terawet bagi tujuan pendidikan perubatan telah menyaksikan perubahan yang sedikit sejak sekian lama. Dengan kemajuan teknologi pendidikan perubatan, mayat manusia terawet masih menjadi pilihan utama untuk mendidik mahasiswa atau pelajar lepasan siswazah dalam kursus-kursus yang berkaitan perubatan dan kesihatan. Di sini kami ingin berkongsi pengalaman kami memulihara mayat terawet secara pembalseman dalam iklim panas dan lembap yang mencabar bagi tujuan pendidikan perubatan melalui pendekatan arteri femoral tunggal tanpa sebarang saluran keluar, menggunakan resipi pembalseman yang mudah dan terbukti berkesan melalui ujian masa.</p>\r\n', 1),
(64126, 64113, 'Akut myeloid leukemia (AML) adalah sub-jenis akut leukemia yang kebiasaannya mempunyai prognosis yang tidak baik. Protein Msi2 merupakan penanda prognostik yang baru yang telah dianggap sebagai sasaran baru untuk terapi AML.Setakat ini, kajian ekpresi Msi2 protein di dalam kes AML belum pernah dijalankan lagi.Tujuan utama kajian ini adalah untuk melihat ekspresi Msi2 protein pada pesakit AML menggunakan  teknik immunohistokimia (IHC) dan  mengaitkan ekspresi tersebut dengan prognostik yang telah dikenalpasti dan parameter klinikal dalam AML beserta survival keseluruhan (OS). Enam puluh empat biopsi trephine tulang sum-sum  telah  melalui proses immunopewarnaan  untuk Msi2 protein. Peratusan  sel blast dengan reaksi positif dan intensiti pewarnaan cytoplasmic dan nuklear telah dinilai.Ekspresi Msi2 protein ditemui di dalam sejumlah 95.3% kes dengan corak Msi2 yang berbeza-beza antara kes.\r\nDalam 71.9% kes, sel blast menunjukkan  positivity selular  secara keseluruhan dan 23.4% kes hanya menunjukkan positivity sitoplasma. Majoriti menunjukkan ekspresi Msi2 yang  tinggi untuk pewarnaan cytoplasmic.Amat menariknya apabila terdapat korelasi yang signifikan antara jumlah pewarnaan selular total dan subkumpulan sitogenetik pertengahan (P = 0.04). Hasil kajian menunjukkan bahawa majoriti daripada pesakit mempunyai ekspresi Msi2 yang tinggi tetapi ia tidak menunjukkan korelasi dengan OS.Walau bagaimanapun, ekspresi Msi2 telah menunjukkan  korelasi yang baik dengan penemuan sitogenetik.Keputusan ini menunjukkan penyelidikan secara meluas pada masa hadapan perlu dijalankan untuk menentukan  dengan lebih tepat peranan sel-sel blast yang positif Msi2 dalam AML di kalangan populasi di Malaysia dan  juga menentukan korelasinya terhadap prognosis and  keseluruhan  kesejahteraan pesakit. \r\n', 1),
(53307, 53295, NULL, NULL),
(53560, 53548, NULL, NULL),
(54252, 54240, '<p>\r\n	Laporan ini menekankan tentang kejadian tindak balas buruk clozapine yang langka i.e. alahan kutaneus dan angioedema visera, pada seorang pesakit skizofrenia yang rintang-rawatan. Kami melaporkan tentang seorang pesakit skizofrenia rintang-rawatan yang mengalami masalah alahan pada kulit dan sistem gastrointestinal sejurus beliau menerima rawatan ubat clozapine. Beliau kemudiannya dirawat dengan kombinasi farmakoterapi namun masih mengalami gejala-gejala sisa. Manifestasi alahan kepada ubat clozapine and strategi perawatan akan dibincangkan di dalam kertas kerja ini. Di sana terdapat keperluan yang mendesak bagi penghasilan ubat psikotropik yang setanding clozapine.</p>\r\n', 1),
(54378, 54366, '<p>\r\n	Peningkatan ekspresi dan aktiviti dan enzim 11&beta;-hidroksisteroid dehidrogenase jenis 1 (11&beta;-HSD1) di dalam sel adiposit matang menyebabkan pembentukan obesiti dan sindrom metabolik. Fruktos di dalam air minuman telah terbukti menyebabkan pembentukan sindrom metabolik pada tikus Wistar jantan. Oleh itu, kajian ini dilakukan untuk melihat kesan ke atas ekspresi dan aktiviti dan enzim 11&beta;-HSD1 di dalam hati pada model tikus sindrom metabolik yang dirangsang oleh air minuman fruktos. Sebanyak 12 ekor tikus Wistar jantan dibahagikan secara rawak kepada dua kumpulan: kumpulan kawalan, C (n=6) dan kumpulan yang diberi minuman fruktos 20%, F20 (n=6). Pengambilan makanan dan air minuman diberikan selama lapan minggu secara ad libitum. Di akhir eksperimen, pengukuran ekspresi enzim 11&beta;-HSD1 di dalam hati dilakukan dengan menggunakan teknik pewarnaan imunohistokimia. Skor pewarnaan diberikan mengikut kepada intensiti granul di dalam sitoplasma hepatosit dengan menngunakan teknik &lsquo;double-blinded&rsquo;. Manakala, aktiviti enzim 11&beta;-HSD1 diukur dengan menggunakan teknik ELISA. Selepas lapan minggu pengambilan air minuman fruktos, kumpulan F20 menunjukkan peningkatan ekspresi dan aktiviti enzim 11&beta;-HSD1 di dalam hati. Data yang diperolehi menunjukkan bahawa enzim 11&beta;-HSD1 di dalam hati mungkin memainkan peranan bagi pembentukan sindrom metabolik dan komplikasinya pada tikus Wistar jantan.</p>\r\n', 1),
(55169, 55156, '<p>\r\n	Ibu bapa kepada kanak-kanak dengan penyakit kronik seperti talasemia mengalami kebimbangan akibat daripada pelbagai tekanan; ibu bapa ini seterusnya akan melalui proses penyesuaian untuk mengatasi situasi ini. Salah satu langkah yang penting adalah mengenalpasti keluarga yang mungkin memerlukan intervensi psikososial agar rawatan perubatan yang holistik dapat disediakan untuk kanak-kanak ini. Satu kajian keratan rentas telah dijalankan untuk menentukan tahap tekanan keibubapaan di kalangan ibu bapa kanak-kanak dengan talasemia. Faktor-faktor risiko yang berkaitan dengan skor tekanan keibubapaan yang tinggi telah dikenal pasti. Instrumen kajian yang digunakan adalah &lsquo;Parenting Stress Index/Short Form&rsquo; dan &lsquo;Coping Inventory for Stressful Situations&rsquo;. Seramai 54 subjek telah direkrut. Jumlah skor tekanan keibubapaan yang tinggi (TPSS, ditakrifkan di sentil ke-75) bagi kohort ini adalah 99.5 manakala min TPSS adalah 86.18. Secara keseluruhan, 24% daripada ibu bapa ini mempunyai TPSS yang tinggi. Bangsa Cina, bilangan anak, pendapatan bulanan, mekanisma pengendalian yang berorientasikan tugasan dan emosi dikaitkan dengan TPSS yang tinggi. Analisa regresi logistik menunjukkan bahawa skor berorientasi tugasan yang rendah tetapi skor berorientasi emosi yang tinggi merupakan faktor-faktor yang mempunyai daya prediktif terbesar. Pengiktirafan awal tekanan keibubapaan adalah penting untuk mengenalpasti ibu bapa yang berisiko agar intervensi seperti kaunseling dan sokongan sosial dapat diberi.</p>\r\n', 1),
(65400, 65387, '<p>\r\n	Mengenalpasti jenis organisma dalam kes jangkitan ulser kornea adalah sangat penting supaya rawatan antibiotik yang sesuai dapat disediakan untuk pesakit. Kaedah konvensional dengan mengunakan kultur plat agar tidak memberi hasil kajian yang memberangsangkan. Percubaan kami dengan teknik yang diubahsuai menggunakan campuran cecair BACTEC untuk mengenalpasti organisma dalam tiga kes jangkitan ulser kornea memberi keputusan cemerlang 100%.</p>\r\n', 1),
(66263, 66250, 'Jangkitan tuberculosis di paru-paru sering berlaku. Akan tetapi, jangkitan tuberculosis extrapulmonari di dalam saluran pernafasan sangat jarang. Manakala, jangkitan tuberculosis di nasofaring adalah teramat jarang. Kami repot 2 kes pembantu kesihatan yang di dapati kelenjar adeniod yang bengkak di nasofaring. Kajian makmal menunjukkan pesakit-pesakit tersebut telah dijangkiti oleh tuberculosis di nasofaring. ', 1),
(67410, 67397, NULL, NULL),
(67412, 67399, NULL, NULL),
(67414, 67401, NULL, NULL),
(67416, 67403, NULL, NULL),
(67484, 67471, 'Keadaan status mental yang rendah di dalam polytrauma merupakan cabaran  kepada pasukan kecemasan dalam pengurusan pesakit. Kecenderungan untuk terlepas penemuan positif dalam keadaan ini meningkat beberapa kali ganda kerana pelbagai faktor seperti keadaan mental status berubah / mabuk, kehadiran kecederaan mengganggu dan kecederaan yang mengancam nyawa serentak yang memerlukan perhatian yang lebih mendesak. Memandangkan ini, NEXUS (National Emergency X-Radiografi Kajian Penggunaan) kriteria menjelaskan  tulang belakang cervical telah diterima pakai dan digunakan di seluruh dunia. Walau bagaimanapun dan sepakat mengenai kebenaran dari bahagian-bahagian lain tulang belakang masih kurang. Laporan kes menonjolkan kes yang hilang dari beberapa patah toraks yang hanya didiagnosis selepas ketibaan di jabatan kecemasan tiga hari kemudian.', 1),
(67534, 29692, '<p>\r\n	Selulitis orbital merupakan jangkitan tisu di sekitar mata di dalam ruangan orbit yang termasuk saraf mata. Ia boleh menyebabkan komplikasi yang membawa kematian sekiranya merebak melalui saraf mata dan ke otak. Penyebab utama jangkitan adalah termasuk perebakan jangkitan sinusitis dari ruangan paranasal atau melalui selulitis preseptal. Kes ini menggambarkan jangkitan di luar kebiasaan mengenai jangkitan orbital selulitis yang berlaku akibat luka torehan pada konjunktiva mata yang disebabkan oleh kemalangan. Rawatan antibiotik sistemik yang agresif mengurangkan risiko komplikasi penglihatan. Kesemua luka pada atau sekelililng mata haruslah dirawat dengan sebaiknya bagi mengelakkan berlakunya komplikasi yang membahayakan. </p>\r\n', 1),
(67548, 67534, '<p>\r\n	Kes ini melaporkan tentang variasi saraf &lsquo;recurrent laryngeal&rsquo; (saraf ke pita suara) yang jarang ditemui, iaitu jenis kedua (Type II) seperti yang pernah diterbitkan oleh Shao et al sebelum ini. Laporan kes ini memfokuskan mengenai seorang wanita berusia 74 tahun yang mempunyai masalah tumbesaran pada kelenjar tiroid yang telah menjalani pembedahan membuang keseluruhan kelenjar tiroid, dan didapati signal saraf yang dipantau hilang secara tiba-tiba semasa pembedahan tersebut. Pembedahan diteruskan menunjukkan kedudukan saraf &lsquo;recurrent laryngeal&rsquo; semasa memasuki ke kawasan pita suara adalah tidak normal. Kecederaan pada saraf tersebut telah dijangka awal, dan seterusnya beliau telah dirujuk kepada pakar bedah bahagian larinks (pita suara) dan didapati beliau mengalami lumpuh pada sebelah pita suara yang berkenaan. Suntikan manipulasi pada pita suara yang terlibat (injection laryngoplasty) telah dilakukan berjaya merawat sementara masalah suara yang terganggu. Beliau kemudian pulih sepenuhnya dalam tempoh enam bulan daripada kelumpuhan pita suara yang sementara.</p>\r\n', 1),
(67602, 67588, '<div>\r\n	Beberapa tahun kebelakangan ini kecenderungan kajian terhadap madu didapati meningkat dengan meluasnya apabila ia telah dikenalpasti mempunyai potensi untuk memberi manfaat kepada kesihatan, pencegahan penyakit dan berfungsi sebagai penggantian perubatan moden. Madu merupakan produk bahan gula semulajadi yang dihasilkan oleh lebah madu dari nektar bunga dan telah digunakan untuk merawat beberapa jenis penyakit seperti penyakit kardiovaskular, kencing manis, kanser, dan penyakit Alzheimer&rsquo;s sejak zaman purba. Madu Gelam merupakan salah satu madu Monofloral liar Malaysia yang sering digunakan secara meluas sebagai bahan perubatan tradisional dalam kalangan penduduk tempatan. Madu Gelam diperkayai dengan beberapa ciri-ciri terapeutik seperti anti-pengoksidaan, anti-radang, anti-kanser dan meningkatkan tahap penyembuhan luka. Walau bagaimanapun, terdapat kekurangan sastera yang memberi tumpuan kepada kesan keseluruhan Madu Gelam dan nilai perubatannya. Kertas kajian ini meringkaskan ciri-ciri fotokimia Madu Gelam berserta dengan nilai-nilai terapeutik yang membawa kepada peningkatan spektrum idea yang luas terhadap produk kesihatan semulajadi. Penjelasan yang ringkas didokumenkan terhadap ciri-ciri terapeutik Madu Gelam untuk memberi pengetahuan baru kepada masyarakat saintifik di dalam era globalisasi &nbsp;moden kini.</div>\r\n', 1),
(67605, 67591, '<div>\r\n	Tulang metatarsal kelima adalah antara tulang kaki yang paling kerap mengalami patah. Pilihan perawatan sama ada melalui pembedahan ataupun konservatif masih dalam perbincangan. Sistem skor, model pilihan analisis dan sistem klasifikasi telah dibentuk untuk membantu membuat pilihan terbaik sama ada perawatan melalui pembedahan ataupun konservatif. Anatomi yang unik dan komplikasi pembedahan mempengaruhi strategi perawatan pembedahan. Oleh itu, artikel ini cuba melihat faktor-faktor yang mempengaruhi perawatan patah tulang metatarsal kelima.</div>\r\n', 1),
(67610, 67596, '<div>\r\n	<div>\r\n		Kecederaan buku lali adalah kejadian yang biasa berlaku dalam mana-mana aktiviti sukan. Tujuan utama kajian adalah untuk menjelaskan anatomi buku lali, mekanisma kecederaan yang berkaitan dengan aktiviti sukan, keabnormalan secara kongenital atau variasi anatomi yang berkaitan dengan kecederaan buku lali serta perbincangan rawatan secara efektif. Suatu tinjauan literatur telah dijalankan untuk mengetahui kecederaan buku lali yang berlaku akibat daripada pelbagai aktiviti sukan. Kami mendokumenkan semua sukan yang melibatkan kecederaan pada sendi buku lali. Anatomi pelbagai struktur bahagian tapak kaki dan keterlibatannya dalam kecederaan dibincangkan dengan panjang lebar. Pengetahuan anatomi tentang kecederaan buku lali boleh memberi manfaat untuk diagnosis akan datang dan bagi tujuan rawatan.</div>\r\n	<div>\r\n		&nbsp;</div>\r\n</div>\r\n', 1),
(67614, 67600, '<div>\r\n	Pembaziran ubat menimbulkan beban kewangan kepada ekonomi negara dan juga, implikasi alam sekitar yang berkaitan dengan pelupusan pembaziran. Kajian ini dijalankan di sebuah hospital awam di Brunei dan bertujuan untuk mengukur tahap pembaziran ubat-ubatan dan pelupusannya. Ia adalah satu kajian rentas/silang bahagian yang menggunakan soal selidik direka sendiri untuk para pesakit mengunjungi farmasi pesakit luar di hospital Suri Seri Begawan (SSB); seramai 253 pesakit mengambil bahagian dalam tempoh dua minggu. Terdapat kriteria pengecualian, iaitu pesakit di bawah usia 18 tahun, pesakit mengunjungi Jabatan Kemalangan dan Kecemasan, dan pesakit yang dimasukkan ke hospital. Majoriti peserta dilaporkan mempunyai ubat yang tidak digunakan di rumah (75.1%, n=189). Hampir separuh (54.2%) mempunyai ubat-ubatan yang tidak digunakan dan ia adalah disebabkan oleh peningkatan dalam keadaan perubatan mereka. Kebanyakkan peserta (70.8%) tidak diberi nasihat yang bersesuaian mengenai cara untuk melupuskan ubat-ubatan mereka dengan cara yang betul. Kebanyakkan mereka membuang ubat-ubatan yang tidak digunakan ke dalam tong sampah di rumah (70%), walaupun 50.2% (n=189) mengetahui bahawa membuang ubat-ubatan dalam tong sampah boleh menyebabkan kesan buruk kepada alam sekitar. &nbsp;Selain itu, didapati bahawa 47.7% daripada peserta akan mengembalikan ubat-ubatan yang tidak digunakan ke farmasi, tetapi secara praktikal, hanya 18.6% telah melakukannya. Tiada hubungan yang signifikan antara mengetahui kesan-kesan memudaratkan perubatan ke atas alam sekitar dan mengembalikan ubat mereka ke farmasi (p=0.065). Kajian ini menunjukkan bahawa wujud pembaziran ubat di hospital SSB dan pengetahuan serta amalan pesakit terhadap pelupusan yang betul adalah agak lemah. Oleh itu, penggubal dasar kesihatan perlu dimaklumkan mengenainya agar dapat melaksanakan strategi pengurangan pembaziran.</div>\r\n', 1),
(67618, 67604, '<p>\r\n	Kerosakan neuron aruhan stress oksidatif yang disebabkan oleh akumulasi hydrogen peroksida (H2O2) dalam sel, telah ditunjukkan terlibat dalam penyakit neurodegeneratif. Beras dan jagung boleh menjadi sumber anti-oksidan dengan kehadiran fitokimia seperti melatonin dan triptofan untuk melawan sepsis reaktif oksigen (ROS). Melatonin telah diketahui boleh meningkatkan ekspresi gen brain-derived neurotrophic factor (BDNF) untuk merangsang fungsi sel saraf dan mengawal-atur kesan anti-penuaan oleh sel otak. Kajian ini bertujuan untuk menentukan kesan beras putih, beras perang, pulut hitam, jagung manis dan &lsquo;baby corn&rsquo; terhadap neurotoksisiti aruhan H2O2 dan mekanisme terlibat dalam sel hipokampus HT22 mencit. Selepas sel HT22 menerima prarawatan ekstrak individu selama 24 jam dan seterusnya menerima cabaran dengan H2O2 selama 24 jam, kebolehhidupan sel, apoptosis, ROS intrasel dan ekspresi gen BDNF diukur. Ekstrak beras dan jagung dinilai untuk aktiviti anti-oksidan dan aras triptofan dan melatonin. Ciri-ciri neurotoksisiti aruhan H2O2 termasuk penurunan kebolehhidupan sel, peningkatan selular ROS dan penurunan ekspresi BDNF. Pra-rawatan dengan ekstrak beras dan jagung mengurangkan secara signifikan neurotoksisiti aruhan H2O2, dan juga mengurangkan secara signifikan pengeluaran ROS. Pra-rawatan dengan extrak-ekstrak tersebut berupaya juga untuk meningkatkan ekspresi mRNA dan protin BDNF yang mungkin disumbang oleh kandungan melatonin dan triptofan. Oleh itu, ekstrak beras dan jagung mungkin melindungi sel HT22 dari neurotoksisiti aruhan H2O2 dengan merencat pengeluaran ROS dan modulasi ekspresi BDNF.&nbsp;</p>\r\n', 1),
(67628, 67614, '<p>\r\n	Menopaus merupakan pemberhentian pendarahan haid, ia secara langsung berkaitan dengan penyakit kardiovaskular (CVD) seperti tekanan darah tinggi dan strok. Penghasilan estrogen dan pengambilan diet berkolesterol tinggi mempunyai kesan yang menjejaskan fungsi kardiovaskular dan metabolisma pada wanita. Kajian ini bertujuan untuk menghasilkan model tikus hipertensi dalam tikus Sprague-Dawley terovariektomi yang diberi makan dengan 2% diet kolesterol pada tempoh jangka pendek. Lapan belas tikus Sprague-Dawley dibahagikan secara rawak kepada tiga kumpulan iaitu kumpulan kawalan Sham (sham), kumpulan kawalan terovariektomi diberi diet normal (Ovx + N), tikus terovariektomi diberi diet kolesterol 2% (Ovx + Cho) selama empat minggu. Pada akhir kajian, pengambilan makanan dan berat badan diukur. Tekanan darah sistolik, diastolik dan tekanan min darah ditentukan. OVX + Cho menunjukkan peningkatan yang ketara dalam perubahan fisiologi seperti berat badan dan pengambilan makanan berbanding dengan kumpulan Ovx + N. Peningkatan yang ketara dalam tekanan darah sistolik, diastolik dan tekanan min darah diperhatikan dalam kumpulan Ovx + Cho. Berdasarkan hasil kajian di atas, ia dipercayai bahawa makanan berkolesterol selama empat minggu (jangka pendek) menyebabkan peningkatan ketara dalam tekanan darah, pengambilan makanan dan berat badan dalam tikus terovariektomi.</p>\r\n', 1),
(67631, 67617, '<div>\r\n	Periferal intravena kateter (PIK) digunakan secara meluas untuk terapi dalam amalan jagarawatan klinikal. Jururawat harus mempunyai pengetahuan yang kukuh dengan amalan jagarawatan dalam pencegahan jangkitan aliran darah dalam kalangan pesakit. Tujuan kajian ini adalah untuk mengenalpasti tahap pengetahuan dan amalan jururawat terhadap penjagaan PIK. Kajian deskriptif keratan rentas yang melibatkan 84 responden telah dijalankan. Soalan selidik terdiri daripada dua domain: 11 item berkaitan dengan pengetahuan dan 15 item amalan kejururawatan. Hasil kajian melaporkan skor yang lebih tinggi dalam kalangan responden unit jagarawatan khas dengan pengetahuan (M = 49.19, SD = 3.44) dan amalan (M = 66.38, SD = 5.15), berbanding dengan responden daripada wad perubatan pembedahan dengan pengetahuan (M = 46.25, SD = 4.68) dan amalan (M = 63.17, SD = 4.63) ke arah penjagaan PIK. Terdapat perbezaan yang signifikan di antara responden daripada unit jagarawatan khas dan wad perubatan pembedahan iaitu pengetahuan (t = -2.641; nilai p = 0.010); dan amalan jagarawatan PIK (t = -2.670, nilai p = 0.009). Terdapat juga perbezaan yang signifikan antara penyelia jururawat dan pengetahuan jururawat berdaftar dalam pengetahuan (t = -3,068; nilai p = 0.003); dan amalan (t = -3.325; nilai p = 0.001) ke arah jagarawatan PIK. Walau bagaimanapun, tidak terdapat perbezaan yang signifikan di antara kelayakan pendidikan, umur dan tahun pengalaman responden terhadap jagarawatan PIK dengan nilai p &gt; 0.05. Kesimpulannya, responden daripada unit jagarawatan khas dan penyelia jururawat boleh disifatkan memiliki pengetahuan yang lebih kukuh dan amalan jagarawatan dalam penjagaan PIK di hospital swasta ini.</div>\r\n', 1),
(67654, 67640, '<div>\r\n	Mutasi telah di kenal pasti berlaku sebelum pembentukan pelbagai jenis kanser. Mutasi di dalam gen p53 boleh di jumpai dalam kanser payudara. Objektif kajian ini adalah untuk menentukan korelasi antara protein p53 dengan wanita dalam usia pra-menopaus. Sebanyak 111 tisu kanser payudara telah dikaji untuk ekspresi protein p53 oleh (Immunohistochemistry) &lsquo;IHC&rsquo;. Hasil kajian mendapati majoriti 36.9.% (n = 41/111) wanita berumur 41 tahun ke atas yang menghidap kanser payudara ekspress &nbsp;lebih p53 protein positif (+) dan hanya 2.7% (n = 3/111) bawah 41 tahun ekspress protein p53 positif (+) berlebihan. Walau bagaimanapun, ujian Fishers Exact menunjukkan bahawa, terdapat hubungan yang tidak signifikan antara kumpulan umur peserta dengan kategori protein p53 (&chi;2 (1) = 0.78; p = 0.52); &nbsp;Cramer&rsquo;s V = 0.08; p = 0.37). Anggaran risiko menunjukkan kebarangkalian ekspresi &nbsp;protein p53 (+) yang melampau dalam kumpulan yang berusia bawah daripada 41 tahun adalah 0.66 kali lebih kurang berbanding kumpulan wanita yang berusia 41 tahun ke atas. Kesimpulannya, dengan atau tanpa ekspresi protein p53 yang melampau, wanita yang berusia lebih daripada 41 tahun mempunyai risiko yang lebih besar untuk &nbsp;menghidap kanser payudara.</div>\r\n', 1),
(67662, 67648, '<div>\r\n	Rawatan konvensional laser argon menyebabkan ketebalan sementara pada lapisan saraf retina (RNFL). Tetapi kesan dari &lsquo;pattern scanning laser&rsquo; (PASCAL) tidak banyak diketahui. Kajian ini dilakukan untuk membandingkan kesan selepas konvensional laser argon dibandingkan dengan PASCAL pada pesakit yag mengalami masalah diabetik retinopati. Sebanyak 32 subjek telah terpilih dalam kajian ini. Seramai 16 pesakit dalam kumpulan argon dan 16 pesakit dalam kumpulan PASCAL. Pesakit yang terlibat diambil dari klinik mata, Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Pemeriksaan mata yang lengkap dan gambar fundus dilakukan pada awal kajian, selepas 2 bulan dan 4 bulan dilaser. RNFL diukur menggunakan domain masa tomografi koheren optikal (OCT). Kedua kumpulan mempunyai ciri klinikal yang sama sebelum rawatan (p= 0.323). RNFL selepas laser argon tidak berubah secara signifikan dalam semua kuadran pada bulan ke 2 dan 4 selepas rawatan. Namun, untuk kumpulan PASCAL, perubahan sigifikan telah ditemui pada bulan ke 4 rawatan pada &lsquo;average RNFL&rsquo; dan RNFL &lsquo;inferior&rsquo; (p&lt;0.05). Bahagian lain menunjukkan kenaikan ketebalan RNFL tetapi tidak signifikan secara statistik. Sebagai kesimpulan, kenaikan RNFL berlaku pada kedua-dua kumpulan. Pesakit PASCAL mengalami lebih ketebalan. Dengan itu, adalah perlu mengambil kira kejadian laser yang baru berlaku jika mahu menganalisa ketebalan RNFL.</div>\r\n', 1),
(67667, 67653, '<div>\r\n	Pencarian bio-penanda telah menjadi agenda utama aktiviti penyelidikan bagi penyakit Rheumatoid Arthritis (RA) pada dekad yang kebelakangan ini. Tujuan kajian ini adalah untuk menentukan korelasi serum matriks metalloproteinase-3 (MMP-3) dengan aktiviti penyakit, kerosakan sendi dan ketidakupayaan pada pesakit dengan RA. Ini adalah satu kajian kes-kawalan keratan rentas yang melibatkan pesakit RA yang berada di bawah rawatan susulan di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Maklumat mengenai ciri-ciri penyakit RA telah diperolehi daripada rekod perubatan dan semua pesakit RA telah dinilai untuk DAS28 (skor aktiviti penyakit berdasarkan 28 sendi) dan Stanford Penilaian Kesihatan Questionnaire (HAQ) 8-item Indeks Upaya (HAQ-DI). Radiograf tangan pesakit RA dinilai dengan menggunakan Skor Modified Sharp (MSS). Serum MMP-3 dari pesakit RA dan kawalan yang sihat diukur dengan menggunakan kaedah ELISA. Seramai 77 pesakit RA dan 18 kawalan sihat telah mendaftar dalam kajian ini. Tahap serum MMP-3 adalah lebih tinggi di kalangan pesakit RA (p &lt;0.05). Terdapat hubungan yang signifikan antara serum MMP-3 dan MSS (r = 0.327) dan HAQ-DI (r = 0.256), kedua-dua p &lt;0.05. Tahap min serum MMP-3 pada pesakit RA dengan hakisan tulang radiografik adalah jauh lebih tinggi daripada pada pesakit tanpa hakisan (p &lt;0.05). Tahap min MMP-3 di kalangan pesakit dengan ketidakupayaan yang signifikan iaitu HAQ-DI &ge;1; jauh lebih tinggi berbanding pesakit RA tanpa kecacatan yang signifikan (p &lt;0.05). Berdasarkan analisis multivariat, HAQ-DI kekal sebagai prediktor serum MMP-3 di kalangan pesakit RA. Serum MMP-3 adalah penanda bio yang berpotensi meramalkan kerosakan sendi radiografik dan ketidakupayaan di kalangan pesakit RA.</div>\r\n', 1),
(67674, 67660, '<div>\r\n	Dua petanda kesihatan salur darah baru yang dihasilkan daripada gelombang fotopletismografi jari (PPG) telah diperkenalkan berdasarkan populasi rakyat Malaysia iaitu indeks kecergasan PPG (PPGF) dan indeks jangkaan risiko salur darah (VRPI). Antara objektif kajian ini adalah untuk mengkaji hubungan antara PPGF dengan petanda penyakit jantung (CVD) yang lain seperti halaju gelombang denyutan karotid femoral (PWVCF), untuk membandingkan PPGF antara mereka yang sihat dan mempunyai faktor risiko CVD dan untuk menentukan sensitiviti VRPI dalam mengenalpasti mereka yang mempunyai faktor risiko CVD. Seramai 114 lelaki berumur antara 20 hingga 40 tahun yang sihat atau mempunyai mempunyai faktor risiko CVD telah diambil. Faktor risiko CVD termasuk hipertensi, merokok, dislipidemia, obesiti abdomen dan sejarah keluarga berpenyakit CVD pramatang. Subjek dibahagikan kepada kumpulan sihat, mereka yang mempunyai satu faktor risiko dan mereka yang mempunyai dua atau lebih faktor risiko. Berat, tinggi, tekanan darah (BP) periferi dan pusat, PWVCF dan PPGF mereka diukur dan sensitiviti VRPI dalam mengenal pasti mereka yang berisiko CVD dikira. Data dianalisis dengan menggunakan SPSS versi 15 dan nilai P&lt;0.05 adalah signifikan. Purata umur subjek adalah 28.94 &plusmn; 4.86 tahun. Tiada perbezaan pada PPGF didapati antara kumpulan-kumpulan (p &gt; 0.05). Pembolehubah tidak bersandar untuk PPGF adalah tekanan hadapan (Beta = 0.35, p &lt; 0.01), PWVCF (Beta = -0.26, p &lt; 0.01), BP sistolik (Beta = -0.26, p = 0.04) dan ketinggian (Beta = 0.24, p &lt; 0.01). Sensitiviti VRPI adalah 82.02%. Kesimpulannya, PPGF adalah berkait dengan PWVCF dan berpotensi untuk menjadi petanda kekenyalan arteri. Tambahan lagi, VRPI adalah sensitif untuk digunakan sebagai petanda untuk saringan awal faktor risiko CVD.</div>\r\n', 1),
(67680, 67666, '<div>\r\n	Kes keracunan plumbum (Pb) yang membimbangkan telah meningkat di beberapa negara tertentu. Semakin banyak bukti kajian menunjukkan pendedahan kronik terhadap plumbum memainkan peranan besar yang menyebabkan ketidakseimbangan prooksidan: antioksidan di dalam tisu otak dan perubahan histologi otak. Kajian ini dilakukan untuk mengenal pasti kesan agen antioksidan daripada kelapa sawit Malaysia, fraksi kaya tokotrienol (TRF) pada tisu otak tikus eksperimen berikutan keracunan plumbum. Lapan belas (n=18) tikus Sprague-Dawley jantan, berusia 6 minggu, dibahagikan secara rawak kepada kumpulan kawalan (CTRL) dan kumpulan eksperimen; didedahkan kepada 0.2% w / v plumbum asetat, sebagai kumpulan PB2; dan yang didedahkan kepada 0.2% w / v plumbum asetat bersama pengambilan suplemen TRF (200 mg / kg berat badan) sebagai kumpulan PB2T. Eksperimen ini dijalankan selama 30 hari. Pada akhir kajian, tisu otak telah diambil dan perubahan histopatologi di kawasan hippocampus diperhatikan. Keputusan biokimia seperti aras plumbum, TRF dan malondialdehid (MDA) dalam tisu otak serta aras aktiviti superoksida dismutase (SOD) dalam eritrosit telah ditentukan. Terdapat neuron atipikal berciri apoptosis dan tidak teratur diperhatikan di kawasan hippocampus bagi kumpulan Pb2 yang didedahkan kepada plumbum berbanding kumpulan Pb2T. Parameter- parameter biokimia tisu otak menunjukkan penurunan aras plumbum yang siginifikan (p&lt;0.05) di dalam kumpulan Pb2T berbanding Pb2. Walaupun tiada perbezaan yang signifikan diperolehi (p&gt;0.05) bagi aras MDA, terdapat peningkatan yang signifikan (p&lt;0.05) bagi aktiviti SOD eritrosit dalam PB2T berbanding PB2 dan CTRL. Pengambilan suplemen TRF dilihat dapat membaiki perubahan histopatologi dalam tisu otak yang disebabkan oleh pendedahan plumbum melalui air minuman dengan mengurangkan pengumpulan plumbum dalam tisu otak tikus eksperimen.</div>\r\n', 1),
(67688, 67674, '<div>\r\n	Polypoidal choroidal vasculopathy (PCV) adalah penyakit retina yang dicirikan oleh aneurismal polip dalam salur darah koroid. Penyakit PCV kerap berlaku dikalangan populasi yang berpigmen terutama di kalangan orang Asia. Kebanyakan laporan mengenai corak penyakit PCV disekitar Asia hanya berdasarkan satu bangsa sahaja (e.g. kaum Cina, Jepun). Amat sedikit laporan mengenai corak penyakit ini dalam populasi pelbagai bangsa seperti di Malaysia. Oleh itu, kajian ini bertujuan menggambarkan ciri-ciri demografi, klinikal dan penyiasatan PCV dalam populasi pelbagai bangsa di Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM). Ini adalah kajian retrospektif pada 91 mata dari 86 pesakit PCV, terdiri daripada Cina (65.1%), Melayu (31.4%), India (2.3%) dan Eurasian (1.2%). Semua pesakit telah menjalani pemeriksaan oftalmik dan penyiasatan terperinci. Purata umur pesakit adalah 70.4 tahun, kebanyakannya lelaki (59.3%) dan majority mengalami masalah didalam satu mata sahaja (94.1%). Purata penglihatan adalah log MAR 0.78 &plusmn; 0.64. Luka-luka &nbsp;vaskular polypoidal umumnya didapati dalam kawasan makular (86.8%) dan menunjukkan pendarahan terutamanya dibawah lapisan makular (59.3%). Menariknya, beberapa mata (43.9%) menunjukkan &quot;drusen&rdquo;. Optikal kepaduan tomografi kebanyakannya menunjukkan perubahan eksudatif (75.9%) dan hampir semua pesakit &nbsp;(97.7%) mempunyai kehilangan membran pengehad external (ELM) dan IS/OS. Dengan ICG angiografi, kebanyakan mata didapati ada lebih dari satu polip (82.4%) dan konfigurasi polip yang paling kerap adalah dalam bentuk &lsquo;kelompok&rsquo; (58.2%). Kesimpulannya, walaupun secara amnya corak PCV di PPUKM sama dengan corak PCV dipopulasi Asia yang lain, namun beberapa pesakit mempunyai &quot;drusen&rdquo;. Oleh itu, PCV di Malaysia mungkin berlainan dengan populasi Asia lain dimana ia mungkin adalah varian degenerasi makular dan tidak idiopatik semata-mata.</div>\r\n', 1),
(67691, 67677, '<div>\r\n	Penggunaan Perubatan Komplementari dan Alternatif (CAM) dalam kalangan pesakit yang mengidap penyakit kronik adalah semakin popular. Kajian ini merupakan kajian keratan rentas deskriptif yang dilakukan di 45 klinik kesihatan kerajaan di Negeri Sembilan. Responden di klinik diabetes dipilih melalui pensampelan rawak bersistematik dan ditemu ramah menggunakan borang soal selidik yang berstruktur. Penggunaan PKA dibahagikan kepada tiga kumpulan: PKA untuk diabetes (CAM-DM), PKA untuk kesihatan umum (CAM-G) dan Bukan Pengguna. Prevalen penggunaan PKA dalam kalangan pesakit diabetes jenis II di Negeri Sembilan adalah 58.5% (CAM-DM: 40.6% dan CAM-G: 17.9%). Bagi kumpulan CAM-DM, peria (Momordicacharantia) merupakan CAM paling popular untuk membantu kawalan diabetes, manakala susu suplemen merupakan pilihan paling popular untuk kumpulan CAM-G. Kesimpulannya, penggunaan CAM dalam kalangan pesakit diabetes jenis II di Negeri Sembilan adalah lazim. Produk semulajadi adalah pilihan utama jenis CAM untuk membantu kawalan diabetes. Kebanyakan pengguna CAM tidak memberitahu anggota kesihatan tentang penggunaan CAM mereka.</div>\r\n', 1),
(67693, 67679, '<p>\r\n	Tomografi berkomputer mikro (mikro-CT) adalah peralatan yang berguna untuk memantau pemulihan patah tulang pada model osteoporosis. Ia menyediakan imej 3D kalus fraktur yang boleh digunakan untuk mengira parameter tulang secara kuantitatif. Dalam kajian in, mikro-CT telah digunakan untuk menilai pemulihan patah tulang tikus yang diorkidektomi, iaitu model osteoporosis defisien-androgen. Kesan ke atas pemulihan patah tulang oleh Eurycoma longifolia, sejenis tumbuhan ubatan dengan aktiviti pro-androgenik, dinilai menggunakan micro-CT. Tikus dibahagikan kepada empat kumpulan: kawalan orkidektomi (ORX), bedah sham (SHAM), orkidektomi dan disuntik dengan testosteron secara intramuskular seminggu sekali (TEN) dan orkidektomi dan menerima oral gavaj Eurycoma longifolia (EL) setiap hari. Jangkamasa rawatan ialah enam minggu. Patah tulang diaruh ke atas tibia kanan kesemua tikus. Mikro-CT telah digunakan untuk mengimbas kalus fraktur yang terdiri daripada 100 potongan aksial di atas dan di bawah aras fraktur. Analisa mikro-CT berjaya mengesan perbezaan signifikan pada pemulihan fraktur kumpulan ORX dan TEN. Bagaimanapun, tiada perubahan mikro-CT yang signifikan dilihat pada pemulihan fraktur kumpulan EL. Kesan EL ke atas pemulihan fraktur tidak dapat dilihat pada model tikus orkidektomi.</p>\r\n', 1),
(67696, 67682, '<div>\r\n	Pengukuran histomorfometri tulang diperlukan untuk memahami kesan penyakit tulang dan juga keberkesanan rawatan pada metabolisme tulang dalam satu tempoh masa. Kajian sebelum ini menggunakan teknik Weibel sebagai kaedah stereologikal kuantitatif untuk menentukan perubahan sel dan dinamik tulang. Walau bagaimanapun, tidak ada penerangan tentang bagaimana teknik ini telah digunakan. Kajian ini bertujuan untuk memberikan gambaran lengkap tentang penggunaan teknik Weibel untuk mengukur indeks histomorfometri tulang statik dan dinamik. Kepakaran teknikal, pemprosesan optimum sampel tulang, perawakan hirisan tisu trabekular dan jumlah imej teranalisa yang mencukupi untuk setiap bahagian adalah diperlukan untuk mencapai keputusan yang boleh dipercayai dengan kemungkinan ralat yang rendah.</div>\r\n', 1),
(67701, 67687, '<div>\r\n	Solar retinopathi merupakan satu penyakit retina yang disebabkan oleh pandangan terhadap cahaya matahari. Diagnosis penyakit ini amat mencabar dan memerlukan sejarah kesihatan dan penyiasatan yang teliti. Laporan kes ini adalah berkenaan seorang pemetik buah-buahan yang berumur 55 tahun yang mengalami kehilangan penglihatan kekal di bahagian tengah medan penglihatan. Diagnosis solar retinopathi telah dibuat berdasarkan kepada sejarah yang diberi oleh pesakit berkenaan, bersama penyiasatan menggunakan pengimejan seperti optical coherence tomography (OCT), infra merah (IF) dan fundus autofluorescence (FAF) yang memainkan peranan penting dalam kes ini. Kesimpulannya, solar retinopathi adalah satu penyakit yang sukar untuk dipulihkan. Penyiasatan dengan cara pengimejan memainkan peranan yang penting untuk merangkul diagnosis yang betul. Justeru itu, pemakaian cermin mata hitam adalah disarankan untuk semua pesakit yang sering terdedah kepada cahaya matahari.</div>\r\n', 1),
(67704, 67690, '<div>\r\n	Skizofrenia merupakan penyakit mental yang kronik dan dicirikan dengan gangguan persepsi, pemirikiran, kognitif dan perubahan tingkah-laku. Salah satu daripada gejala utama skizofrenia ialah halusinasi auditori. Di dalam laporan ini, kita membincangkan kes seorang lelaki pelarian Rohigya yang berumur &nbsp;31 tahun yang telah mengamputasikan zakar beliau secara total akibat dari halusinasi arahan yang diterima oleh beliau, di mana beliau telah pun sakit sejak sekian lama di Myanmar. Beliau telah dikejarkan ke Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) untuk mendapatkan rawatan. Setelah keadaan beliau stabil, beliau telah dimasukkan ke wad psikiatri untuk pemerhatian dan rawatan perubatan selanjutnya. Beberapa isu berkaitan dengan factor psikososial, ekonomi dan politik menjadikan kes ini unik untuk dibincangkan. Penama telah dirawat dengan ubat sulpride dan telah didiscaj ke pihak pembedahan urologi untuk rawatan selanjutnya.</div>\r\n', 1),
(67708, 67694, '<div>\r\n	Trakeostomi boleh dikaitkan dengan pelbagai komplikasi. Di sini, kami membentangkan kes berkaitan penjagaan tiub trakeostomi yang tidak sempurna di mana seorang pesakit telah menggunakan tiub trakeostomi melebihi jangkamasa tanpa menukarnya menyebabkan komplikasi tisu parut kulit yang besar pada trakeostoma dan tisu parut di dalam trakea yang menyebabkan kedudukan tiub trakeostomi teralih dari kedudukan asal seterusnya mengakibatkan halangan pada salur pernafasan. Kes ini menggambarkan bahawa kesedaran dan pengetahuan berkaitan penjagaan tiub trakeostomi yang betul di kalangan kakitangan perubatan adalah sangat penting bagi mengelakkan sebarang salah urus yang boleh mengakibatkan komplikasi yang mengancam nyawa kepada pesakit.</div>\r\n', 1),
(67715, 67701, '<div>\r\n	Stenosis bukaan piriform nasal kongenital (CNPAS) adalah penyebab yang amat jarang ditemui untuk kesempitan saluran udara hidung. Secara klinikal, ia menyerupai atresia choanal pada bayi neonate. Ia perlu dibezakan kerana pengurusan kedua-dua penyakit ini adalah berbeza. Bukaan piriform hidung merupakan bahagian yang paling hadapan dan paling sempit di saluran udara hidung. Oleh itu, sekiranya berlaku sedikit kesempitan di kawasan rentas hidung, ia akan menyebabkan hidung tersumbat. CNPAS biasanya dikaitkan dengan kecacatan pembangunan garis tengah seperti kecacatan holoprosensefali atau pituitari. Kami membentangkan kes yang pertama dilaporkan dalam kesusateraan saintifik bahasa Inggeris, CNPAS yang berkaitan dengan Trisomi 8 Mosaicsm (T8M).</div>\r\n', 1),
(67717, 67703, '<div>\r\n	Kepatahan tulang boleh terjadi selepas serangan sawan. Walaupun ia jarang berlaku, kelewatan dalam diagnosa dan rawatan dapat dielakkan sekiranya pemeriksaan fizikal yang teliti dilakukan terhadap golongan yang berisiko tinggi. Di sini, kami melaporkan satu kes yang mana kami hampir terlepas mendiagnosa kepatahan di bahagian proksimal humerus selepas serangan serangan sawan tonik-klonik. Seorang wanita berusia 58 tahun yang menghidap penyakit epilepsi telah datang ke Jabatan Kecemasan kerana mengadu sakit badan setelah mendapat serangan sawan tonik-klonik yang berlaku beberapa jam sebelumnya. Semasa pemeriksaan, beliau didapati berada dalam kesakitan serta tidak berupaya untuk menggerakkan kesemua anggota badannya. Selepas diberikan ubat tahan sakit, beliau masih mengadu berasa sakit serta tidak boleh menggerakkan kedua-dua bahunya. Pemeriksaan lanjutan menunjukkan pergerakan yang terhad dalam semua arah pada kedua-dua bahu beliau namun tiada abnormaliti kelihatan pada struktur bahunya. X-ray pada kedua-dua bahagian bahu beliau menunjukkan kepatahan pada bahagian proksimal tulang humerus kiri dan kanan. Kepatahan bahagian proksimal tulang humerus jarang berlaku dengan sendirinya berikutan sawan; mereka selalu dikaitkan dengan dislokasi sendi glenohumeral.&nbsp;</div>\r\n', 1),
(67723, 67709, '<div>\r\n	Cytomegalovirus (CMV) Retinitis merupakan penyakit yang berlaku pada pesakit yang menghidapi Human Immunodeficiency Virus (HIV) terutamanya. Ia juga dilaporkan pada pesakit tanpa HIV, contohnya pesakit menghidapi gangguan autoimun yang memerlukan immunosuppresif, pesakit menjalani pemindahan organ atau sumsum tulang, pesakit kanser darah atau payudara yang menerima kemoterapi, penyakit mata berikutan suntikan kortikosteroid intraokular atau periokular, kencing manis dan Good sindrom. Walau bagaimanapun, kes CMV retinitis dalam pesakit yang menghidapi dermatomyositis dan kanser pada masa yang sama tidak pernah dilaporkan sebelum ini. Ia juga tidak dilaporkan dalam kanser selain kanser darah atau payudara, atau pesakit kanser sebelum menerima rawatan kemoterapi. Kami melaporkan kes CMV retinitis dalam seorang wanita tanpa HIV berusia 40 tahun yang menghidapi dermatomyositis dan kanser paru-paru, di mana CMV retinitisnya kembali apabila immunosuppresif dimula semula. Kedua-dua episod CMV retinitis telah berjaya dirawat selepas mengambil kira keadaan keimunannya.</div>\r\n', 1),
(67727, 67713, '<p>\r\n	-</p>\r\n', 1),
(67733, 67719, 'Kekurangan zat besi adalah punca utama penyakit anemia. Kajian ini dibuat untuk menilai kelaziman anemia dan kekurangan zat besi dalam wanita mengandung dalam populasi Malaysia dan hubung kaitnya dengan socio-demografi dan profil obstetrik. Kajian keratan rentas dibuat di klinik kesihatan bandar dalam jangka masa enam bulan. Satu sampel darah diambil daripada wanita mengandung yang kelihatan sihat pada lawatan pertama antenatal dan dihantar ke makmal untuk penilaian makmal termasuk pengiraan darah lengkap dan feritin serum sebagai alat saringan untuk anemia dan status zat besi. SPSS versi 19.0 diguna untuk analisis statistik. Daripada sejumlah 250 subjek, 43.6% mengidap anemia dan 31.6% mengidap kekurangan zat besi. Sejumlah besar iaitu 47.7% daripada subjek yang mengidap anemia juga mengalami kekurangan zat besi, manakala 19.1% daripada subjek yang kekurangan zat besi tidak pula mengalami anemia. Ferritin serum mempunyai korelasi negatif dengan kematangan kandungan pada lawatan pertama antenatal (p<0.001), dengan seramai 77.6% daripada kumpulan wanita ini tidak mengambil zat besi tambahan sebelum itu. Feritin serum juga kekurangan antara wanita yang beranak lebih daripada lima (p=0.01). Kekurangan zat besi juga sangat ketara (p=0.024) dalam kalangan kaum India (42.5%) berbanding dengan kaum Melayu (33.5%) dan Cina (13.0%). Kesimpulannya, oleh kerana ramai wanita mengandung mengalami kekurangan zat besi, bukan sahaja yang mengalami anemia tetapi juga yang mempunyai nilai hemoglobin normal, kesinambungan praktis memberi tambahan zat besi kepada semua wanita mengandung di Malaysia adalah wajar.\r\n', 1),
(67750, 67736, '<p>\r\n	Leptospirosis merupakan penyakit berjangkit yang semakin banyak dilaporkan di serata dunia. Simptom-simptom penyakit ini adalah tidak spesifik dan hampir menyerupai simptom jangkitan lain seperti deman denggi, malaria, hepatits dan deman kepialu, terutamanya di negara-negara tropika di mana penyakit-penyakit tersebut adalah endemik. Demam denggi pula adalah penyakit berjangkit yang dilihat sebagai krisis kesihatan awam disebabkan kadar jangkitan sedunia yang tinggi. Dalam laporan kes ini, kami melaporkan satu kes berkenaan seorang pesakit yang menghidapkan demam denggi yang juga disahkan positif dengan jangkitan leptospirosis melalui ujian serologi. Koinfeksi antara demam denggi dan leptospirosis boleh menimbulkan kerumitan pada peringkat diagnosis kerana simptom-simptom bagi kedua-dua jangkitan tersebut hampir serupa. Oleh itu, pakar klinikal harus mempunyai indeks wasangka yang tinggi,khususnya di kawasan di mana kedua-dua penyakit ini adalah endemik. Penggunaan ujian diagnosis yang berasaskan antigen adalah amat penting bagi membantu pakar klinikal dalam membuat diagnosis yang tepat secepat mungkin agar rawatan yang sewajarnya dapat diberikan.</p>\r\n', 1),
(67824, 67810, NULL, NULL),
(67829, 67815, 'Di Malaysia, timun laut lebih dikenali sebagai Gamat. Di kalangan kaum Melayu, gamat sering digunakan sebagai ubat tradisional untuk melegakan kesakitan, merawat luka dan kesan terbakar. Ianya juga digunakan sebagai tonik untuk memberi sumber tenaga tambahan.Stichopus chloronotus merupakan salah satu spesies timun laut yang boleh didapati di Malaysia. Kajian ini dilakukan untuk penentuan dos optimum ekstrak akues Stichopus chloronotus emulsi salap ke atas luka pada model tikus. Beberapa siri kepekatan iaitu 0.1%, 0.5% dan 1% ekstrak akues Stichopus chloronotus emulsi salap diberikan ke atas luka eksisi sekali sehari selama 10 hari. Perubahan pada kawasan luka diukur dengan menggunakan angkup dan gambar luka diambil pada hari ke-1, ke-3, ke-6, ke-8 dan ke-10 selepas pembentukan luka. Keputusan dariapada peratusan pengurangan luka dan pemerhatian makroskopik akan menentukan dos optimum Stichopus chloronotus. Hasil kajian menunjukkan, kumpulan tikus kajian yang menerima rawatan Stichopus chloronotus 0.5% (SC 0.5%) mempunyai peratusan pengurangan luka yang lebih tinggi dan pemerhatian makroskopik yang lebih baik bermula dari hari ke-6 selepas pembentukan luka berbanding kumpulan yang lain. Kesimpulannya dos 0.5% merupakan kepekatan optimum bagi Stichopus chloronotus memberikan kesan kepada penyembuhan luka dan akan digunakan pada kajian sebenar.', 1),
(67857, 67843, 'Adenoid cystic carcinoma yang berasal daripada septum hidung merupakan penyahit yang jarnag ditemui. Kanser ini walaupun mampu dikawal dengan perubatan optimum pada awalnya, mampu merebak ke bahagian lain badan bertahun-tahun kemudian. Faktor-faktor prognostik telah dikenalpasti and implikasinya adalah penting dari segi kaunseling pesakit dan pilihan terapi. Kita memaparkan satu kes adenoid cystic carcinoma yang berasal daripada setum hidung dengan invasi perineural. Kanser ini telah dibuang secara endoskopik dan pesakit ini telah menerima radioterapi adjuvant.', 1),
(67859, 67845, 'Ligasi arteri sphenopalatine merupakan satu terapi surgikal untuk masalah hidung berdarah. Prosedur ini memerlukan pengetahuan yang luas mengenai variasi anatomi hidung. Walaubagaimanapun, petanda yang biasanya dipercayai seperti crista ethmoidalis mungkin tidak dijumpai atau tiada langsung. Variasi sekian adalah penting and boleh memutuskan keberkesanan prosedur ini. Kita memaparkan satu kes variasi foramen dan arteri sphenopalatine yang tiada crista ethmoidalis.', 1),
(67862, 67848, 'Fistula branchial lengkap dengan bukaan luaran dan dalaman adalah satu fenomena yang jarang ditemui. Biasanya, ia dihubung-kait dengan masalah radang kelenjar tiroid dan bengkak bernanah bahagian leher. Dengan tanda-tanda yang berkenaan, doktor pakar bedah boleh meminta pelbagai ujian untuk mendapatkan diagnosis. Ia termasuk imbasan tomografi berkomputer dengan kontras atau pengimejan resonans bermagnet bahagian leher. Kami memaparkan kes di mana satu ujian mudah iaitu fistulogram yang telah dilakukan mampu memberikan diagnosis fistula branchial lengkap.', 1),
(67992, 67978, 'Madu gelam telah didapati mempunyai kesan anti-oksidatif, anti-kanser dan anti-inflamasi terhadap banyak jenis kanser. Tujuan kajian ini dalah untuk menentukan kesan madu gelam terhadap aras tekanan oksidatif sel kanser peparu manusia. IC50 madu gelam ditentukan dengan merawat sel A549 dengan dos madu yang berbeza (50-150 mg/ml). Sel dibahagikan kepada 4 kumpulan dan diaruh tekanan oksidatif dengan menggunakan hidrogen peroksida (H2O2) mengikut kumpulan tertentu: kawalan, H2O2, madu gelam, H2O2 + madu gelam. Selepas 24 jam rawatan, biopetanda tekanan oksidatif seperti malondialdehid (MDA) dan protein karbonil ditentukan. Aruhan tekanan oksidatif meningkatkan aras MDA (p<0.05) tetapi tidak memberi kesan kepada protein karbonil. Rawatan madu gelam didapati menurunkan aras MDA (p<0.05) dalam sel A549 yang diaruh dengan H2O2 tetapi tidak memberi kesan kepada protein karbonil. Rawatan madu gelam sahaja tidak mempunyai kesan terhadap aras MDA dan protein karbonil. Kesimpulannya, madu gelam menurunkan peroksidasi lipid tetapi bukan oksidasi protein dalam sel peparu manusia teraruh tekanan oksidatif.', 1),
(67996, 67982, 'PENGENALAN : Terapi Oksigen Hiperbaric (TOH)  dikesan dapat meningkatkan pengoksigenan dan kesan anti-mikrob yang berpotensi meningkatkan penyembuhan ulser kronik. Kajian ini adalah bertujuan untuk menilai kesan TOH dalam ulser kaki diabetik  (UKD) yang kronik.\r\nKaedah: Seramai enam puluh pesakit dikelaskan mengikut Wagner 1, 2 atau 3 ulser kaki diabetik kronik, telah diambil dan kemudiannya dibahagikan secara rawak kepada dua kumpulan; TOH dan kumpulan kawalan. Semua pesakit menjalani rawatan standard untuk UKD, tetapi bagi kumpulan TOH yang, menjalani 20 sesi TOH, yang berlangsung selama 80 - 90 minit pada 2.5 atmosfera mutlak (ATA) setiap sesi. Bilangan sel putih dan tahap protein C-reaktif telah diambil semasa kemasukan wad, pada minggu kedua dan keempat rawatan. Saiz luka didokumentasikan pada setiap susulan sehingga enam bulan susulan. Soal selidik SF-36 selepas pesakit menjalani terapi oksigen hiperbarik telah digunakan untuk mengukur kualiti kehidupan dan tahap kesihatan.\r\nKeputusan: Enam puluh responden yang berumur antara 39-77 tahun, di mana seramai 31 pesakit lelaki (51.2%) dan 29 (48.3%) pesakit wanita telah dipilih. Pengurangan sel putih dan protin C-reaktif dalam kumpulan TOH adalah ketara sepanjang rawatan berbanding kumpulan kawalan (p = 0.046 dan p = 0.039 masing-masing). 26 pesakit (86.7%) daripada kumpulan TOH telah mencapai penyembuhan ulser pada enam bulan susulan, manakala hanya 18 pesakit (60%) dalam kumpulan kawalan telah sembuh sepenuhnya pada enam bulan susulan. Pesakit yang dirawat dengan TOH menunjukkan konstituen kesihatan mental dan fizikal jauh lebih baik dalam soal selidik kualiti kehidupan\r\nKesimpulan: Adalah ditekankan bahawa TOH adalah terapi tambahan kepada rawatan ulser kaki diabetik yang kronik dalam  mempercepatkan penyembuhan luka bagi memastikan kualiti hidup pesakit yang lebih baik.\r\n\r\n', 1),
(68044, 68030, 'Hiperkalemia sederhana selalunya tidak menyebabkan gejala yang melibatkan jantung. Akan tetapi jika ia berlaku dan seiring dengan penggunaan ubat yang merencat fungsi atrio-venrikular (AV), ia boleh membawa musibah yang berat buat pesakit terutama golongan tua. Disini kami melaporkan sebuah kes bradiarithmia berpanjangan disebabkan hiperkalemia yang terjadi disebabkan rawatan perubatan bersama penggunaan ubat yang merencat fungsi atrio-ventriukular. Seorang wanita berusia 81 tahun yang menerima rawatan atas beberapa masalah perubatan telah datang dengan gejala bradiarithmia. Tahap potassium beliau adalah sederhana tinggi dan fungsi buah pinggang juga terjejas.  Gejala bradikardia beliau berpanjangan dan beliau bergantung kepada ubat isoproterenol sehingga fungsi buah pinggang kembali normal. Gejala bradiarithmia mungkin akan berlaku kepada pesakit golongan tua didalam situasi dimana berlaku hiperkalemia disebabkan kerosakan fungsi buah pinggang didalam pernggunaan ubat yang merencat fungsi atrio-ventrikular. Lebih lagi didalam pesakit yang mengambil pelbagai ubat-ubatan.', 1);
INSERT INTO `content_field_abstrak` (`vid`, `nid`, `field_abstrak_value`, `field_abstrak_format`) VALUES
(68063, 68049, 'Amplikasi isoterma berpengantara gelung (LAMP) merupakan teknik amplifikasi gen yang menghasilkan produk akhir iaitu mendapan keruh magnesium pirofosfat yang boleh  dianalisis dengan hanya menggunakan mata kasar. Penggunaan pewarna interkalat yang sesuai adalah penting kerana ia boleh meningkatkan sensitiviti dan mengurangkan keputusan positif palsu dan negatif palsu untuk pengesanan. Kajian ini bertujuan untuk membandingkan prestasi tiga pewarna interkalat yang berbeza; SYBR Green I, SYBR Safe dan pewarna berasaskan calcein di dalam asai LAMP HPV-16 melalui visualisasi oleh mata kasar, elektroforesis gel dan mesin masa-nyata turbidimeter. Reaksi LAMP dilakukan menggunakan amplifikasi kit Loopamp DNA berisipadu sebanyak 25 µl. Campuran reaksi dieram pada suhu 60°C selama 60 minit dan ditamatkan menggunakan suhu 80°C selama 5 minit dalam mesin masa-nyata turbidimeter. Untuk pengesanan menggunakan mata kasar, SYBR Green I dan SYBR Safe telah dicairkan dalam 1:10 DMSO dan telah ditambah ke dalam tiub yang mengandungi campuran reaksi selepas proses amplifikasi berlaku manakala calcein ditambah sebelum proses amplifikasi. Sensitiviti asai telah disiasat menggunakan pencairan DNA HPV-16 yang berkepekatan bermula dari 101 salinan/µl to 108 salinan/µl. Ketiga-tiga pewarna mempamerkan keputusan yang sama dari segi sensitiviti dengan had pengesanan adalah 103 salinan/µl. Penambahan calcein di dalam asai menunjukkan masa pengesanan bertambah selama 10 minit dengan menggunakan mesin nyata-masa turbidimeter. Prestasi ketiga-tiga pewarna interkalat untuk pengesanan mata kasar adalah setanding dan boleh digunakan untuk aplikasi pemeriksaan titik akhir dalam asai HPV-16, manakala dengan mesin nyata-masa turbidimeter, penambahan calcein melambatkan masa pengesanan selama 10 minit.', 1),
(68069, 68055, 'Artritis septik pinggul kanak-kanak adalah kejadian serius yang boleh membawa kesan jangka panjang yang tidak diingini. Untuk neonat, ia mengakibatkan keadaan pinggul yang teruk  terutamanya jika rawatan lambat dimulakan. Antara akibat daripada artritis septik ini termasuk kerosakan epifisis yang menjurus kepada kecacatan anggota badan, degenerasi sendi lebih awal dan limitasi pergerakan sendi. Pembedahan pada waktu yang tepat boleh memperbaiki keadaan dan fungsi pinggul. Dislokasi pinggul akibat sepsis juga boleh berlaku namun terdapat segelintir pesakit yang terlibat dengan dislokasi ini didapati mempunyai kepala femur yang elok. Baru-baru ini, kumpulan pesakit sebegini dianggap sebagai entiti tersendiri. Laporan kes ini menerangkan tentang seorang kanak-kanak berumur 2 tahun yang berjaya dirawat selepas dislokasi pinggul septik dan menunjukkan keputusan yang baik 11 tahun kemudiannya.\r\n\r\n', 1),
(68143, 68129, 'Angiosarcoma adalah sejenis sarkoma yang jarang berlaku. Ia adalah agresif, ketumbuhan malignan sel endothelial yang berasal daripada vaskular atau limfa. Angiosarcoma menyumbang  2% daripada semua sarkomas dan lebih separuh daripadanya berlaku di bahagian kepala dan leher. Rawatannya adalah mencabar dengan prognosis yang tidak baik. Di sini kami membincangkan sebuah kes Angiosarcoma daripada inferior turbinate yang berlaku kepada seorang lelaki berusia 73 tahun.', 1),
(68147, 68133, 'ABSTRAK\r\nAkut myeloid leukemia (AML) adalah sub-jenis akut leukemia yang kebiasaannya mempunyai prognosis yang tidak baik. Protein Msi2 merupakan penanda prognostik yang baru yang telah dianggap sebagai sasaran baru untuk terapi AML.Setakat ini, kajian ekpresi Msi2 protein di dalam kes AML belum pernah dijalankan lagi.Tujuan utama kajian ini adalah untuk melihat ekspresi Msi2 protein pada pesakit AML menggunakan  teknik immunohistokimia (IHC) dan  mengaitkan ekspresi tersebut dengan prognostik yang telah dikenalpasti dan parameter klinikal dalam AML beserta survival keseluruhan (OS). Enam puluh empat biopsi trephine tulang sum-sum  telah  melalui proses immunopewarnaan  untuk Msi2 protein. Peratusan  sel blast dengan reaksi positif dan intensiti pewarnaan cytoplasmic dan nuklear telah dinilai.Ekspresi Msi2 protein ditemui di dalam sejumlah 95.3% kes dengan corak Msi2 yang berbeza-beza antara kes.\r\nDalam 71.9% kes, sel blast menunjukkan  positivity selular  secara keseluruhan dan 23.4% kes hanya menunjukkan positivity sitoplasma. Majoriti menunjukkan ekspresi Msi2 yang  tinggi untuk pewarnaan cytoplasmic.Amat menariknya apabila terdapat korelasi yang signifikan antara jumlah pewarnaan selular total dan subkumpulan sitogenetik pertengahan (P = 0.04). Hasil kajian menunjukkan bahawa majoriti daripada pesakit mempunyai ekspresi Msi2 yang tinggi tetapi ia tidak menunjukkan korelasi dengan OS.Walau bagaimanapun, ekspresi Msi2 telah menunjukkan  korelasi yang baik dengan penemuan sitogenetik.Keputusan ini menunjukkan penyelidikan secara meluas pada masa hadapan perlu dijalankan untuk menentukan  dengan lebih tepat peranan sel-sel blast yang positif Msi2 dalam AML di kalangan populasi di Malaysia dan  juga menentukan korelasinya terhadap prognosis and  keseluruhan  kesejahteraan pesakit. \r\n\r\n\r\n', 1),
(68157, 68143, NULL, NULL),
(68253, 68239, '‘Basaloid squamous cell carcinoma’ (BSCC) adalah variasi ‘squamous cell carcinoma’ yang jarang berlaku dan agresif yang kebanyakannya ditemui dalam saluran pernasafan atas. BSCC jarang ditermui di tonsil. BSCC telah sering ditemui di kelejar limfa (64%) dan metastasis (44%). Tiada dokumen berkaitan BSCC di kepala dan leher dilaporkan melalui carian PubMed 2004-2014. Kes kami adalah megenai seorang lelaki berusia 46 tahun dengan BSCC tonsil, T2N0M0; dan pada awalnya telah terkawal melalui pembedahan dan radioterapi. 3 tahun kemudian, beliau mempunyai metastasis melibatkan jari tengah kiri. ‘Ray pemotongan’ jari tersebut dilakukan sebagai langkah rawatan paliatif. ', 1),
(68352, 68338, 'Plasmasitoma ekstramdulari adalah komplikasi myeloma berbilang yang sangat jarang berlaku. Kami melaporkan kes seorang wanita berumur 56 tahun yang menghidap myeloma berbilang yang telah mengalami bengkak pada mata kiri. Ia menyebabkan bola mata beliau tersembul, luka pada kornea dan juga kemerosotan penglihatan. Biopsi tisu menunjukkan perebakan plasma sel neoplastik yang menepati cir-ciri plasmasitoma. Penurunan bengkak yang ketara pada mata kiri jelas kelihatan setelah beliau melalui proses radio-kemoterapi. Walaubagaimanapun, kemerosotan penglihatan tidak menunjukkan sebarang perubahan.', 1),
(68353, 68339, 'Selulitis orbital merupakan jangkitan tisu di sekitar mata di dalam ruangan orbit yang termasuk saraf mata. Ia boleh menyebabkan komplikasi yang membawa kematian sekiranya merebak melalui saraf mata dan ke otak. Penyebab utama jangkitan adalah termasuk perebakan jangkitan sinusitis dari ruangan paranasal atau melalui selulitis preseptal. Kes ini menggambarkan jangkitan di luar kebiasaan mengenai jangkitan orbital selulitis yang berlaku akibat luka torehan pada konjunktiva mata yang disebabkan oleh kemalangan. Rawatan antibiotik sistemik yang agresif mengurangkan risiko komplikasi penglihatan. Kesemua luka pada atau sekelililng mata haruslah dirawat dengan sebaiknya bagi mengelakkan berlakunya komplikasi yang membahayakan.', 1),
(68374, 68360, 'Kadar kematian dan kecederaan disebabkan oleh  senjata api telah meningkat secara mendadak kerana kemajuan pengeluaran senjata api dan keadaan yang mudah untuk mendapatkannya. Tujuan kajian ini dijalankan adalah untuk menentukan corak kematian disebabkan oleh senjata api di Malaysia dari tahun 1998 sehingga 2008. Sampel kajian terdiri daripada 52 kes kematian yang disebabkan oleh  tembakan yang diterima oleh Unit Forensik, PPUKM antara tahun 1998 dan 2008. Parameter yang digunakan adalah bangsa, jantina, umur, bentuk jenis luka, jenis senjata, cara kematian, sejarah kes, jarak tembakan, darah untuk toksikologi, penentuan cara kematian dan corak pengedaran luka. Hasil kajian menunjukkan perubahan yang berubah-ubah dalam tempoh kajian ini, mangsa lelaki mencatat 96% daripada kes kematian ini dengan lelaki yang berketurunan cina menunjukkan bilangan mangsa yang tertinggi (56%). Kematian yang disebabkan oleh pembunuhan juga mencatat insiden yang tinggi iaitu 94.2%, dan senjata pistol (57.1%) adalah senjata yang paling kerap digunakan. Pelbagai tempat  tembakan yang disasarkan ditemui di 36 kes pembunuhan dengan (73.5%) di kepala (22%), dada (24.5%), tulang belakang (12.3%), perut (6.2%) dan anggota badan (24.5%). Penemuan kajian autopsi ini diharapkan dapat menyokong fakta fenomena umum kes pembunuhan dan bunuh diri disebabkan oleh senjata api di Malaysia.', 1),
(68506, 68492, NULL, NULL),
(68512, 65387, 'Mengenalpasti jenis organisma dalam kes jangkitan ulser kornea adalah sangat penting supaya rawatan antibiotik yang sesuai dapat disediakan untuk pesakit. Kaedah konvensional dengan menggunakan kultur plat tidak memberi hasil kajian yang memberangsangkan. Percubaan kami dengan teknik yang diubahsuai menggunakan  campuran cecair BACTEC untuk mengenalpasti  organisma dalam tiga kes jangkitan ulser kornea memberi keputusan cemerlang 100%.	', 1),
(68546, 68531, 'Plasma sel myeloma telah diketahui menyebabkan pengeluaran  immunoglobulin (Ig) monoklonal yang mengakibatkan rembesan homogen protein monoklonal (komponen M) yang unik. Walau bagaimanapun, terdapat kes-kes melaporkan bahawa ia juga boleh menyebabkan pengeluaran dua klon protein M monoklonal yang berbeza. Walaupun ia agak jarang berlaku dan hanya 2% kes direkodkan daripada semua kes plasma cell myeloma, ciri-ciri klinikal adalah sama dengan mana-mana kes monoklonal tersebut. Berkemungkinan 2 klon protein M yang berbeza ini adalah hasil dari klon yang sama atau dua klon sel monoklonal yang berbeza. Mana-mana pun mekanisme penyakit ini, tindak-balas terhadap rawatan adalah sama dengan kes-kes monoklonal walaupun beberapa laporan menunjukkan rintangan terhadap rawatan boleh berlaku. Di sini, kami melaporkan satu kes plasma cell myeloma yang jarang direkodkan dimana kes ini menunjukkan penghasilan dua klon protein M yang berbeza iaitu IgG (lambda) dan IgA (lambda), ciri-ciri klinikal, ciri-ciri hematologi serta penanda biokimia dan tindak balas kepada rawatan.', 1),
(68586, 68571, 'Proses penyembuhan luka, terutamanya pembentukan semula epitelium, adalah dikawalatur oleh pelbagai sitokin, kemokin dan faktor-faktor pertumbuhan yang terdapat di kawasan luka. Gangguan dalam pembentukan semula epithelium, selalunya terjadi pada luka kronik dan semasa penuaan kulit, adalah disebabkan oleh kekurangan faktor-faktor penting ini. Rembesan fibroblas yang boleh dikumpul dari media buangan sebagai media terkondisi dari fibroblast kulit (Dermal Fibroblast Conditioned Medium; DFCM) mengandungi faktor-faktor yang boleh mempercepatkan proses pembentukan semula epithelium. Tujuan kajian semasa adalah untuk menjelaskan kesan DFCM ke atas pembentukan semula epithelium oleh keratinosit dari kumpulan umur yang berbeza (≥18-35 tahun, 36-54 tahun dan ≥55 tahun). DFCM telah disediakan daripada pengkulturan fibroblas di dalam media tanpa serum spesifik keratinosit (DFCM-KM) dan spesifik fibroblas (DFCM-FM). Keratinosit yang dikultur dengan tambahan DFCM diperhatikan menggunakan pengimejan selang-masa untuk menilai pelekatan dan kadar pertumbuhan, manakala teknik pencalaran dilakukan untuk menilai kadar penyembuhan luka. Pemerhatian menunjukkan, tanpa mengira umur penderma, DFCM-KM meningkatkan pelekatan keratinosit manakala DFCM-FM meningkatkan kadar penyembuhan luka keratinosit. Penambahan DFCM-KM tidak memberi kesan peningkatan ke atas kadar pertumbuhan, dimana penambahan DFCM-FM mengurangkan kadar pertumbuhan keratinosit secara signifikan. Keputusan ini menunjukkan potensi aplikasi DFCM dalam kes-kes pembentukan semula epithelium yang terjejas.', 1);

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(399, 396, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	In early pregnancy the placenta may attach low in the uterus and covers a part or all of the entrance to the cervix. This attachment can be seen in early ultrasound scans and called a low-lying placenta. In most cases, when the growing uterus enlarges, the placenta is carried upwards and majority does not cause any problem. However some remain in the lower segment (10%) of the uterus to be called placenta praevia (RCOG 2005).</p>\r\n<p>\r\n	The predisposing factors to placenta praevia are multiple gestation (Francois et al. 2003) previous uterine scar (caesarean and abortion) (Ananth et al. 1997), increased parity, uterine structure anoma-lies, increased age (more than 35 years old), previous history of placenta praevia, infertility treatment, smoking (Chelmow et al. 1996) and low socio-economic status (Sheiner et al. 2001). Placenta praevia may present with painless vaginal bleeding during pregnancy or other problem such as abnormal lie. Moreover, in chronic cases patients may have anaemia and present with anaemic symptoms resulting from chronic bleeding (Lam et al. 2000).</p>\r\n<p>\r\n	There are known complications of placenta praevia such as sudden vaginal bleeding, risk of caesarean section, pre-term labour, prone to a repeat placenta praevia in the next pregnancy, as well as hysterectomy with life threatening bleeding, risk in developing morbidly adherent pla-centa (Oyelese &amp; Smulian 2006) in future pregnancies to the extent of maternal and fetal mortality (Hamisu et al. 2003).</p>\r\n<p>\r\n	Due to possibility of such complications, it requires most of these patients to be admitted to a hospital with adequate facilities as a prevention and anticipation to possible problems. However, this is not always easy and not possible as the women and her family may not com-prehend the medical consequences of her problem. Due to the paucity of data avail-able on the knowledge of placenta praevia, this study was carried out to evaluate the knowledge and attitude of obstetric patients regarding placenta praevia.</p>\r\n<p>\r\n	<strong>METHODS</strong></p>\r\n<p>\r\n	A cross sectional study was carried out in Hospital Ipoh, Perak Darul Ridzuan from 26th March to 15th April 2007. All the antenatal and postnatal patients in class 2 and 3 obstetric wards in Ipoh Hospital were included in the study. Patients from labour room and high dependency ward were excluded. There was a total of 323 respondents during the survey. Data was collected manually by using purposive or conducive sampling method with a non-probability sampling.</p>\r\n<p>\r\n	Definition:</p>\r\n<table border="0" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td colspan="4" style="width:271px;">\r\n				<p>\r\n					1. Level of knowledge</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width: 111px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Good&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="2" style="width: 160px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					respondents answer at least 50% questions re-garding knowledge of pla-centa praevia correctly.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width: 111px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Poor&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="2" style="width: 160px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					respondents answer less than 50% questions re-garding knowledge of pla-centa praevia correctly.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="4" style="width:271px;">\r\n				<p>\r\n					2. Educational level</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width: 111px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Primary&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="2" style="width: 160px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					UPSR, equivalent, or be-low</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width: 111px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Secondary :</p>\r\n			</td>\r\n			<td colspan="2" style="width: 160px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					STPM, matriculation, equivalent or below</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width: 111px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Tertiary&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="2" style="width: 160px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					University, Colleges or equivalent</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="4" style="width:271px;">\r\n				<p>\r\n					3. Occupation</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="3" style="width: 119px; height: 47px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Professional&nbsp; (white collar):</p>\r\n			</td>\r\n			<td style="width: 152px; height: 47px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					those who perform tasks which are less &lsquo;physically laborious&rsquo;. Such as doctors, airline pilots, IT professional, or lawyer</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="3" style="width: 119px; height: 46px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Non professional (blue collar):</p>\r\n			</td>\r\n			<td style="width: 152px; height: 47px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					those who perform ma-nual labour. Such as factory workers, building and constructional trades, mechanical work, maintenance or technical installation.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="4" style="width:271px;">\r\n				<p>\r\n					4. Level of Attitudes</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width: 109px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Good&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="3" style="width: 162px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					respondents answer at least 50% questions re-garding positive attitude of placenta praevia correctly.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width: 109px; vertical-align: top;">\r\n				<p style="margin-left:.25in;">\r\n					&middot;Poor&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; :</p>\r\n			</td>\r\n			<td colspan="3" style="width: 162px; vertical-align: top;">\r\n				<p style="margin-left:-5.4pt;">\r\n					respondents answer less than 50% questions re-garding positive attitude of placenta praevia correctly.</p>\r\n			</td>\r\n		</tr>\r\n		<tr height="0">\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	Socio demographic parameters (ie age, race, parity, occupation, educational level), history of placenta praevia were studied in relation to knowledge and attitude towards placenta praevia. The collected data was analysed by statistical analysis and cate-gory variables were done with chi square-test. A p value of &lt;0.05 was considered to be statistically significant. &nbsp;</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	There was a total of 323 respondents included in the study. Out of these, twenty women (6.2%) had current or previous history of placenta praevia. Seventeen (5.3%) came to hospital with presence of placenta praevia. Most of the respondents (82.4%) were less than 35 years old. The majority of the women were Malay (65.6%), followed by Chinese (15.5%), Indian (14.2%) and others (Table 1). Most women were multiparae(52.9%) and were housewives (45.8%). The majority was educated until secondary school (76.5%) with a minimal from primary school (9.6%).</p>\r\n<p>\r\n	Among women with placenta praevia, the prevalence was highest amongst Malays (80%). Sixty percent of those with placenta praevia were multiparae while the remain-ing were grandmultiparae (20%) and primi-gravidae (20%). Increased age was the main predisposing factor found among patients with placenta praevia followed by multiparty, previous caesarean, previous history of placenta praevia and infertility treatment (Figure 1). All women with pla-centa praevia had caesarean section (100%). Eight had history of vaginal bleeding during current or previous preg-nancy. Pre-term labour complicated three women with placenta praevia and only one had postpartum haemorrhage. There were no fetal mortality and morbidity (Figure 2).</p>\r\n<p>\r\n	The level of knowledge among obstetric patients&nbsp;&nbsp; was&nbsp;&nbsp; distributed&nbsp;&nbsp; almost&nbsp;&nbsp; evenly among those with good (48.9%) or poor (51.1%) knowledge. There were five women (25%) in the placenta praevia group with poor knowledge (i.e. less than two correct answers for four questions). The questions were: the need of haematinics during pregnancy, prohibition of sex during pregnancy, the need to admit to hospital when advised and the need to remain in hospital for a long period. Fifteen patients (75%) had good knowledge. Eightwomen (40%) had poor attitude as they ignored at least two of the four advices given, while 12 of them (60%) followed (<em>p</em>value = 0.037).</p>\r\n<p>\r\n	There was a significant relationship between patient&rsquo;s educational backgrounds to the level of knowledge regarding placenta praevia (Table 2). With a history of placenta praevia in their previous pregnancy, their knowledge regarding the same problem had an increment, as shown in Table 2. There was also a significant difference between occupation and level of knowledge regarding placenta praevia (Table 3), as the knowledge was higher among those of &lsquo;white collar&rsquo; jobs as compared to &lsquo;blue collar&rsquo; and housewives. There was a significant relationship between increased age and incidence of placenta praevia (Table 1) as it was higher amongst those of age 35 years old and above (<em>p</em>&lt;0.05).</p>\r\n<p>\r\n	However there were several non-significant findings during the study on obs-tetric patients in Hospital Ipoh including no relationship between level of knowledge and maternal age (<em>p</em>=0.54), parity (<em>p</em>=0.91), race (<em>p</em>=0.052)and complication of placen-ta praevia (<em>p</em>=0.79).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The increasing incidence of caesarean sections and maternal age globally predis-posed more to the incidence of&nbsp; placenta praevia in the obstetric population (Ikechebelu &amp; Onwusulu, 2007). The prevalence of placenta praevia in the current&nbsp; study was higher&nbsp; (6.2%) than seen in previous statistics of Hospital Ipoh (2002-1.4%, 2003-1.26%, 2004-1.23%, 2005-0.91%).This might be due to short duration of the study with smaller number of patients&nbsp; including the exclusion of the first class and high dependency ward patients. Malay women were seen to be the majority having placenta praevia which was likely due to more Malay women being obstetric clients in Hospital Ipoh.</p>\r\n<p>\r\n	As shown in the current and previousstudy several predisposing factors were recognised for placenta praevia, which were multiparity, previous uterine scar (caesarean section), increasing maternal age, previous history of placenta praevia, and infertility treatment (Faiz et al. 2003). In this study, the commonest predisposing factor of placenta praevia was increasing maternal age (30%, 6 cases), followed by multiparity (20%, 4 cases) as shown in Figure 1. All of the respondents with placenta praevia in the study had caesa-rean section (20 out of 20 patients with placenta praevia) with a range of compli-cations (Figure 2) as had been reported by other studies (Anath et al. 1997) with no other detrimental complication of hyste-rectomy as experienced by others (Choi et al. 2008).</p>\r\n<p>\r\n	There was slightly more obstetric patients with poor knowledge regarding placenta praevia (ie mean score of knowledge is 11.18, full marks of the knowledge are 23). This might be due to most of the patients in obstetric wards of Hospital Ipoh have low to moderate educational background as shown in Table 2. Also, Hospital Ipoh receives patients from suburban areas like Grik, Chemor, Sungai Siput and aborigine settlement.</p>\r\n<p>\r\n	The knowledge of placenta praevia was affected by current or previous history of placenta praevia. According to the study, patients with current or previous history of placenta praevia had higher knowledge compared to those who did not have history of placenta praevia. Seventy five percent of those women with current or previous history of placenta praevia had good knowledge while only 47.2% of patients without history of placenta praevia had good knowledge about placenta praevia (Table 2). This may be contributed to the counseling by doctors when they were diagnosed to have placenta praevia or from their previous experience. The women who had placenta praevia also took the initiative to know more about placenta praevia by surfing the internet, reading books and medical articles.</p>\r\n<p>\r\n	The background level of patients&rsquo; education also affected the knowledge about placenta praevia. It was seen that those who had a higher educational status had higher knowledge about placenta praevia (Table 2) which may due to a higher interest level with extra knowledge, as they understood more readily.</p>\r\n<p>\r\n	The knowledge of placenta praevia was also related to the occupation of the respondents. White-collar workers have higher knowledge about placenta praevia than blue-collar workers and housewives (<em>p</em>=0.018). Some &lsquo;white-collar&rsquo; workers came from the medical field like nurses, paramedics who were more accessible to information about placenta praevia.</p>\r\n<p>\r\n	Most respondents in the study with placenta praevia who had good knowledge about placenta praevia also had good attitude for preventing the complications, which included admission for a longer duration in a well equipped hospital once diagnosed to have placenta praevia. This was practiced in other countries as re-ported earlier (Love et al. 2004). Those who had higher knowledge of placenta praevia realised the danger of its com-plications. Compliance to doctor&rsquo;s ad-vice (eg prohibition of sexual intercourse and compliance to haematinics) and medication to ensure their health and fetuses. All had further support from their husbands ren-dering them to have a harmonious mind despite the long stay in hospital. Expe-riencing complications of placenta praevia did not show significance to good know-ledge most likely because only a small proportion had experienced the problem.</p>\r\n<p>\r\n	In general further education and expo-sure on placenta praevia with its possible complications should be emphasised to ob-stetric patients, to ensure proper attitude towards the medical advice given and be compliant to medication in order to achieve optimum care for women with placenta praevia. Therefore, efforts to improve the knowledge regarding placenta praevia through quality improvement programs are very important in order to prevent avoid-able complications such as fetal mortality and maternal death secondary to uncon-trolled bleeding.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The knowledge and attitude towards placenta praevia among the obstetric patients in Hospital Ipoh was better in those who had higher level of education, in &lsquo;white-collar&rsquo; occupation and those with current history of placenta praevia.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	We would like to express our sincere grati- tude to the Director of Hospital Ipoh, Head of Department of O&amp;G of Hospital Ipoh, Dr. Mukudan, all the doctors from O&amp;G department especially Dr. Zeity and other medical staff in Hospital Ipoh for their contributions to this study.</p>\r\n', 2),
(346, 343, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Central&nbsp; corneal&nbsp; thickness&nbsp; plays&nbsp; a&nbsp; major role in the management of many types of glaucoma.&nbsp; Ocular&nbsp; hypertensive&nbsp; (OHT) studies&nbsp; (Kass&nbsp; et&nbsp; al.&nbsp; 2002,&nbsp; Gordon&nbsp; et&nbsp; al. 2002)&nbsp; has&nbsp; not&nbsp; only&nbsp; reported&nbsp; thin&nbsp; cornea as&nbsp; a&nbsp; predictor&nbsp; for&nbsp; the&nbsp; development&nbsp; of primary&nbsp; open&nbsp; angle&nbsp; glaucoma&nbsp; (POAG) but Copt et al (1999) has also reclassified more&nbsp; than&nbsp; 56%&nbsp; of&nbsp; OHT&nbsp; patients&nbsp; based on&nbsp; the&nbsp; corrected&nbsp; central&nbsp; corneal thickness&nbsp; (CCT).&nbsp; Additionally,&nbsp; Kim&nbsp; and Chen&nbsp; (2004)&nbsp; demonstrated&nbsp; progression of&nbsp; disease&nbsp; was&nbsp; more&nbsp; likely&nbsp; in&nbsp; POAG patients&nbsp; with&nbsp; thinner&nbsp; corneas,&nbsp; hence giving&nbsp; another&nbsp; insight&nbsp; in&nbsp; the&nbsp; role&nbsp; of&nbsp; CCT in&nbsp; managing&nbsp; glaucoma&nbsp; patients. Correlation&nbsp; between&nbsp; corneal&nbsp; thickness and&nbsp; the&nbsp; severity&nbsp; of&nbsp; the&nbsp; disease&nbsp; in&nbsp; the POAG&nbsp; group&nbsp; has&nbsp; been&nbsp; described&nbsp; by many&nbsp; authors&nbsp; using&nbsp; advanced&nbsp; glaucoma intervention study (AGIS) scoring (Kim &amp; Chen&nbsp; 2004,&nbsp; Herndon&nbsp; et&nbsp; al.&nbsp; 2004).&nbsp; However,&nbsp; this&nbsp; relationship&nbsp; is&nbsp; not&nbsp; well described for the NTG group of patients.&nbsp;</p>\r\n<p>\r\n	Optical&nbsp; coherence&nbsp; tomography&nbsp; shows good&nbsp; topographic&nbsp; relationship&nbsp; between structural&nbsp; damage&nbsp; and&nbsp; functional&nbsp; loss (visual&nbsp; field&nbsp; loss)&nbsp; (Wollstein&nbsp; et&nbsp; al.&nbsp; 2004, Wollstein et al. 2005). The average RNFL thickness&nbsp; was&nbsp; found&nbsp; to&nbsp; have&nbsp; the&nbsp; strong- est&nbsp; correlation&nbsp; with&nbsp; severity&nbsp; of&nbsp; the&nbsp; dis- ease,&nbsp; followed&nbsp; by&nbsp; the&nbsp; inferior&nbsp; and&nbsp; supe- rior&nbsp; quadrant&nbsp; measurements. The&nbsp; aim&nbsp; of this&nbsp; study&nbsp; was&nbsp; to&nbsp; investigate&nbsp; a&nbsp; possible correlation&nbsp; between&nbsp; the&nbsp; severity&nbsp; of&nbsp; glau- coma&nbsp; (based&nbsp; on&nbsp; RNFL&nbsp; thickness&nbsp; as measured&nbsp; by&nbsp; OCT)&nbsp; and&nbsp; CCT&nbsp; among NTG and POAG group of patients.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS </strong></p>\r\n<p>\r\n	<em>Patients&nbsp; </em></p>\r\n<p>\r\n	An&nbsp; observational&nbsp; study&nbsp; of&nbsp; patients&nbsp; who were treated for primary open angle and normotensive&nbsp; glaucoma&nbsp; was&nbsp; carried&nbsp; out at&nbsp; UKMMC,&nbsp; Kuala&nbsp; Lumpur&nbsp; from&nbsp; January 2006&nbsp; to&nbsp; April&nbsp; 2007.&nbsp; This&nbsp; study&nbsp; was&nbsp; ap- proved&nbsp; by&nbsp; the&nbsp; Medical&nbsp; Research&nbsp; and Ethics Committee UKM.&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<em>Design&nbsp; </em></p>\r\n<p>\r\n	Medical&nbsp; records&nbsp; of&nbsp; the&nbsp; patients&nbsp; were&nbsp; re- viewed&nbsp; retrospectively&nbsp; for&nbsp; selection&nbsp; of cases. Sample size was calculated using the sample size calculation program ver- sion&nbsp; 2.1.30&nbsp; February&nbsp; 2003.&nbsp; High&nbsp; tension glaucoma&nbsp; was&nbsp; diagnosed&nbsp; if&nbsp; the&nbsp; baseline IOP&nbsp; prior&nbsp; to&nbsp; treatment&nbsp; was&nbsp; more&nbsp; than 21mmHg&nbsp; with&nbsp; the&nbsp; presence&nbsp; of&nbsp; open&nbsp; an- gle&nbsp; on&nbsp; gonioscopy&nbsp; and&nbsp; glaucomatous optic&nbsp; disc&nbsp; changes.&nbsp; Those&nbsp; with&nbsp; baseline readings less than 21mmHg were classi- fied&nbsp; as&nbsp; having&nbsp; normal&nbsp; tension&nbsp; glaucoma (NTG).&nbsp; All&nbsp; patients&nbsp; had&nbsp; glaucomatous visual&nbsp; field&nbsp; defects&nbsp; as&nbsp; confirmed&nbsp; by&nbsp; the Humphrey&nbsp; Visual&nbsp; Field&nbsp; test&nbsp; and&nbsp; only&nbsp; pa- tients&nbsp; who&nbsp; were&nbsp; on&nbsp; medical&nbsp; treatment with&nbsp; no&nbsp; previous&nbsp; surgery&nbsp; were&nbsp; recruited for&nbsp; this&nbsp; study.&nbsp; Patients&nbsp; with&nbsp; significant anterior&nbsp; segment&nbsp; disease&nbsp; causing&nbsp; poor signal&nbsp; strength&nbsp; on&nbsp; the&nbsp; OCT&nbsp; were&nbsp; ex- cluded&nbsp; from&nbsp; the&nbsp; study.&nbsp; Other&nbsp; exclusion criteria&nbsp; were&nbsp; those&nbsp; with&nbsp; diabetic&nbsp; retino- pathy, contact lens user and high myopia of more than&nbsp; -&nbsp; 6 dioptre. The&nbsp; worse eye was chosen for the study.&nbsp;</p>\r\n<p>\r\n	Measurement of CCT was done using a specular&nbsp; microscope&nbsp; SP-3000P&nbsp; (Topcon Corporation, Tokyo). This method used a non&nbsp; contact&nbsp; technique&nbsp; to&nbsp; measure&nbsp; the corneal thickness, which was reported to have&nbsp; better&nbsp; repeatability&nbsp; compared&nbsp; to ultrasound&nbsp; pachymetry&nbsp; (Bovelle&nbsp; et al. 1999).&nbsp; Three&nbsp; readings&nbsp; with&nbsp; a standard deviation&nbsp; of&nbsp; less&nbsp; than&nbsp; 3&micro;m&nbsp; were&nbsp; used, and&nbsp; the&nbsp; average&nbsp; was&nbsp; calculated&nbsp; for&nbsp; the analysis.&nbsp; Subsequently,&nbsp; measurement&nbsp; of RNFL was done by a single operator us- ing&nbsp; the Stratus&nbsp; 3 OCT&nbsp; (Carl&nbsp; Zeiss&nbsp; Medi- tech,&nbsp; Dublin,CA)&nbsp; on well-dilated&nbsp; eyes. Good&nbsp; signal&nbsp; strength&nbsp; of&nbsp; 7&nbsp; or&nbsp; more&nbsp; was included&nbsp; for&nbsp; analysis.&nbsp; Diagnosis&nbsp; was confirmed&nbsp; by&nbsp; looking&nbsp; at their&nbsp; previous record&nbsp; of&nbsp; baseline&nbsp; IOP&nbsp; before&nbsp; treatment and patients who were treated elsewhere with&nbsp; no known&nbsp; pretreatment&nbsp; IOP&nbsp; were excluded&nbsp; from&nbsp; the study.&nbsp; Purpose&nbsp; and details of the study were explained to the patient&nbsp; and informed&nbsp; consent&nbsp; was&nbsp; ob- tained.&nbsp; Demographic&nbsp; data,&nbsp; past&nbsp; medical history&nbsp; and&nbsp; ocular&nbsp; history&nbsp; were&nbsp; obtained from all patients.</p>\r\n<p>\r\n	The&nbsp; worse&nbsp; eye&nbsp; was&nbsp; chosen&nbsp; based&nbsp; on the&nbsp; humphrey&nbsp; visual&nbsp; field.&nbsp; Eyes&nbsp; with counting&nbsp; finger&nbsp; or&nbsp; worse&nbsp; vision&nbsp; were&nbsp; ex- cluded.&nbsp; Visual&nbsp; acuity&nbsp; using&nbsp; the&nbsp; logMar chart&nbsp; was&nbsp; recorded&nbsp; and&nbsp; autorefraction was done to look at the level of refractive error. Anterior segment examination was performed&nbsp; and&nbsp; intraocular&nbsp; pressure&nbsp; was measured&nbsp; before&nbsp; gonioscopy&nbsp; was&nbsp; done. All patients were dilated and examination of&nbsp; the&nbsp; retina&nbsp; was&nbsp; performed&nbsp; to&nbsp; look&nbsp; for the&nbsp; presence&nbsp; of&nbsp; other&nbsp; ocular&nbsp; pathology which&nbsp; may&nbsp; affect&nbsp; the&nbsp; RNFL&nbsp; measure- ment.&nbsp; Optical&nbsp; coherence&nbsp; tomography was&nbsp; done&nbsp; for&nbsp; all&nbsp; patients&nbsp; with&nbsp; a&nbsp; dilated pupil&nbsp; and&nbsp; only&nbsp; those&nbsp; with&nbsp; a&nbsp; good&nbsp; signal strength were included in the study. Data was&nbsp; then&nbsp; collected&nbsp; and&nbsp; those&nbsp; with&nbsp; in- complete&nbsp; documentation&nbsp; were&nbsp; excluded. Data of the patients were then analyzed.&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS&nbsp; </strong></p>\r\n<p>\r\n	A&nbsp; total&nbsp; of&nbsp; 190&nbsp; eyes&nbsp; from&nbsp; 190&nbsp; patients were&nbsp; included&nbsp; in&nbsp; the&nbsp; study.&nbsp; There&nbsp; were 60&nbsp; patients&nbsp; in&nbsp; the&nbsp; normal&nbsp; tension&nbsp; glau- coma&nbsp; (NTG)&nbsp; group,&nbsp; 61&nbsp; patients&nbsp; for&nbsp; pri- mary&nbsp; open&nbsp; angle&nbsp; glaucoma&nbsp; (POAG) group&nbsp; and&nbsp; 69&nbsp; patients&nbsp; for&nbsp; the&nbsp; normal (control)&nbsp; group.&nbsp; Age&nbsp; for&nbsp; all&nbsp; the&nbsp; groups were normally distributed (Shapiro-Wilk p &gt;&nbsp; 0.05).&nbsp; Parametric&nbsp; statistical&nbsp; tests&nbsp; were used&nbsp; for&nbsp; the&nbsp; data&nbsp; analyses.&nbsp; All&nbsp; p-values were 2-sided and were considered statis- tically&nbsp; significant&nbsp; when&nbsp; the&nbsp; values&nbsp; were less than 0.05.</p>\r\n<p>\r\n	Mean&nbsp; age&nbsp; for&nbsp; the POAG&nbsp; group&nbsp; was 64.5&plusmn;10.2&nbsp; years,&nbsp; NTG&nbsp; group&nbsp; 65.0&plusmn;7.6 years&nbsp; and&nbsp; the normal&nbsp; group&nbsp; 61.6&plusmn;8.2 years&nbsp; old&nbsp; (One&nbsp; way&nbsp; ANOVA,&nbsp; p=0.195). Intraocular&nbsp; pressure&nbsp; was&nbsp; significantly different&nbsp; between&nbsp; the&nbsp; three&nbsp; groups (p&lt;0.0001)&nbsp; (Table&nbsp; 1).&nbsp; Central&nbsp; corneal thickness of the NTG group was found to be thinnest among the three groups. Sig- nificant&nbsp; differences&nbsp; were&nbsp; noted&nbsp; between the&nbsp; CCT&nbsp; of&nbsp; the NTG&nbsp; patients&nbsp; compared to the control group (p &lt; 0.05) (Table 1).</p>\r\n<p>\r\n	RNFL&nbsp; thickness&nbsp; was&nbsp; thinner&nbsp; in&nbsp; both glaucoma&nbsp; groups&nbsp; compared&nbsp; to&nbsp; the&nbsp; con- trol&nbsp; group&nbsp; of&nbsp; patients&nbsp; (p&lt;0.05)&nbsp; (Table&nbsp; 2). Both&nbsp; glaucoma&nbsp; groups&nbsp; had&nbsp; similar&nbsp; level of severity of disease based on RNFL (p &gt;0.05).</p>\r\n<p>\r\n	A&nbsp; significant&nbsp; Pearson&nbsp; correlation&nbsp; coeffi- cient&nbsp; was&nbsp; found&nbsp; in&nbsp; the&nbsp; POAG&nbsp; group&nbsp; be- tween the CCT and RNFL in the superior quadrant and average thickness (r=0.265, p&lt;0.05 and r=0.417, p&lt;0.05 respectively). No&nbsp; significant&nbsp; correlation&nbsp; was&nbsp; found between the CCT and RNFL for the NTG group (Table 3, Figures 1, 2).&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION&nbsp; </strong></p>\r\n<p>\r\n	Normotensive&nbsp; glaucoma&nbsp; was&nbsp; long&nbsp; consi- dered&nbsp; as&nbsp; a&nbsp; subset&nbsp; of&nbsp; POAG,&nbsp; sharing many&nbsp; similar&nbsp; characteristics&nbsp; except&nbsp; for the&nbsp; elevated&nbsp; IOP&nbsp; (Sowka&nbsp; 2005).&nbsp; In&nbsp; this study, mean CCT for NTG patients were found to be the thinnest compared to the POAG and the control groups. However, based&nbsp; on&nbsp; published&nbsp; calculations, (Doughty &amp; Zaman 2000) the differences&nbsp;&nbsp; were too small to influence the IOP mea- surement&nbsp; in&nbsp; a&nbsp; clinically&nbsp; meaningful&nbsp; way. Therefore&nbsp; the&nbsp; measured&nbsp; IOP&nbsp; achieved using&nbsp; the&nbsp; &#39;gold&nbsp; standard&#39;&nbsp; technique&nbsp; of Goldmann&nbsp; tonometry&nbsp; may&nbsp; not&nbsp; differ significantly&nbsp; from&nbsp; the&nbsp; actual&nbsp; IOP&nbsp; in&nbsp; NTG patients.&nbsp; However,&nbsp; POAG&nbsp; patients&nbsp; were found&nbsp; to&nbsp; have&nbsp; relatively&nbsp; comparable corneal&nbsp; thickness&nbsp; with&nbsp; the&nbsp; normal population&nbsp; supporting&nbsp; the&nbsp; fact&nbsp; that&nbsp; the disease&nbsp; is&nbsp; truly&nbsp; an&nbsp; effect&nbsp; of&nbsp; an&nbsp; ageing trabecular meshwork. The value for CCT&nbsp; in this study however was slightly lower compared&nbsp; to other&nbsp; studies&nbsp; which&nbsp; mainly used&nbsp; ultrasound&nbsp; pachymetry&nbsp; to&nbsp; measure the CCT (Bechmann et al. 2000).&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Significant correlation between the CCT and severity of RNFL was only found for the&nbsp; POAG&nbsp; group&nbsp; but&nbsp; not&nbsp; for&nbsp; the NTG group&nbsp; of&nbsp; patients.&nbsp; The&nbsp; results&nbsp; obtained were&nbsp; also&nbsp; comparable&nbsp; with&nbsp; a cross&nbsp; sec- tional&nbsp; study&nbsp; done&nbsp; using&nbsp; AGIS&nbsp; scoring which did not find any significant correla- tion&nbsp; between&nbsp; the&nbsp; corneal&nbsp; thickness&nbsp; and severity&nbsp; of&nbsp; the disease&nbsp; in&nbsp; NTG&nbsp; patients (Bechmann&nbsp; et al. 2000).&nbsp; The&nbsp; different findings&nbsp; between&nbsp; these&nbsp; two&nbsp; groups&nbsp; of patients&nbsp; may&nbsp; implicate&nbsp; the&nbsp; possibility&nbsp; of different pathophysiology of the disease.</p>\r\n<p>\r\n	&nbsp;Cioffi&nbsp; and&nbsp; Liebman&nbsp; (2002)&nbsp; proposed possible&nbsp; relationship&nbsp; between&nbsp; a&nbsp; thin&nbsp; cor- nea&nbsp; and&nbsp; intracellular&nbsp; matrix&nbsp; changes&nbsp; at the&nbsp; level&nbsp; of&nbsp; lamina&nbsp; cribosa&nbsp; may&nbsp; predis- pose&nbsp; to&nbsp; glaucomatous&nbsp; changes.&nbsp; This idea would be an additional risk factor for this group of patients. Despite controlling the&nbsp; IOP,&nbsp; progression&nbsp; may&nbsp; seem&nbsp; to&nbsp; be inevitable&nbsp; in&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; thin&nbsp; cor- nea. However, this possibility may not be applicable to the NTG patients as similar correlation between the CCT and severity of the disease was not found. Therefore, consideration&nbsp; of&nbsp; NTG&nbsp; as&nbsp; a&nbsp; subset&nbsp; of POAG&nbsp; has to&nbsp; be&nbsp; evaluated&nbsp; with caution. Although&nbsp; clinically&nbsp; the&nbsp; appearance&nbsp; on gonioscopy&nbsp; may&nbsp; be&nbsp; similar,&nbsp; further&nbsp; eval- uation&nbsp; at&nbsp; the&nbsp; cellular&nbsp; and&nbsp; genetic&nbsp; level may&nbsp; help&nbsp; to&nbsp; answer&nbsp; these&nbsp; questions. Other&nbsp; influential&nbsp; factors&nbsp; that&nbsp; may&nbsp; contri- bute to the severity of the disease for the NTG&nbsp; group&nbsp; were&nbsp; proposed&nbsp; by&nbsp; Doyle (Doyle et al. 2005). Higher association of systemic&nbsp; disease&nbsp; in the NTG&nbsp; group may affect&nbsp; the ocular&nbsp; perfusion&nbsp; indirectly leading&nbsp; to optic&nbsp; nerve&nbsp; damage&nbsp; in this group of patients (Doyle et al. 2005).</p>\r\n<p>\r\n	To date, there has been no study done to&nbsp; look&nbsp; at&nbsp; the&nbsp; association&nbsp; of&nbsp; CCT&nbsp; and severity&nbsp; of&nbsp; glaucoma&nbsp; in&nbsp; the&nbsp; NTG&nbsp; group using the OCT. However, this study&nbsp; was a&nbsp; cross&nbsp; sectional&nbsp; study&nbsp; and&nbsp; the&nbsp; stability of&nbsp; the&nbsp; disease&nbsp; was&nbsp; not&nbsp; analyzed.&nbsp; As such, the possibility of further RNFL pro- gression&nbsp; was&nbsp; not&nbsp; taken&nbsp; into&nbsp; considera- tion.&nbsp; A&nbsp; prospective&nbsp; long&nbsp; term&nbsp; study&nbsp; may give a better understanding of the corre- lation between the corneal thickness and RNFL.&nbsp;</p>\r\n<p>\r\n	<strong>CONCLUSION&nbsp; </strong></p>\r\n<p>\r\n	Significant&nbsp; correlation&nbsp; between&nbsp; the&nbsp; cor- neal&nbsp; thickness&nbsp; and&nbsp; severity&nbsp; of&nbsp; glaucoma is only present in POAG patients but not for&nbsp; NTG&nbsp; cases.&nbsp; Our&nbsp; results&nbsp; suggest&nbsp; that CCT&nbsp; is&nbsp; related&nbsp; to&nbsp; the&nbsp; severity&nbsp; of&nbsp; POAG- related&nbsp; visual&nbsp; loss&nbsp; thus&nbsp; measuring&nbsp; the corneal thickness in this group of patients may&nbsp; help&nbsp; to&nbsp; determine&nbsp; which&nbsp; patient would&nbsp; benefit&nbsp; from&nbsp; close&nbsp; monitoring. Furthermore,&nbsp; the&nbsp; old&nbsp; consideration&nbsp; of NTG&nbsp; as&nbsp; a&nbsp; subset&nbsp; of&nbsp; POAG&nbsp; may&nbsp; not&nbsp; be applicable&nbsp; with&nbsp; many&nbsp; studies&nbsp; showing dissimilar&nbsp; detail&nbsp; characteristics&nbsp; between those two groups.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS </strong></p>\r\n<p>\r\n	&nbsp;The&nbsp; authors&nbsp; would&nbsp; like&nbsp; to&nbsp; express&nbsp; their greatest&nbsp; gratitude&nbsp; to&nbsp; the&nbsp; Department&nbsp; of Ophtalmology&nbsp; for&nbsp; the&nbsp; continous&nbsp; support throughout&nbsp; the&nbsp; study,&nbsp; Prof&nbsp; Dr&nbsp; Ropilah Abdul&nbsp; Rahman,&nbsp; Dr&nbsp; Fam&nbsp; Han&nbsp; Borr&nbsp; from Singapore&nbsp; National&nbsp; Eye&nbsp; Centre&nbsp; and&nbsp; Dr Nor Fariza Ngah from Hospital Selayang for their critical comments on this study.</p>\r\n', 1),
(398, 395, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The incidence of intracranial complications of chronic otitis media (COM) is estimated to be between 0.02% and 1.97% (Osama et al 2000). The extracranial and intra-cranial complications occur when chronic infection within the middle ear and mastoid spaces extends to the region beyond the bony confines. Osama et al (2000) review-ed 2890 cases of chronic otitis media from 1990-1999 and found that 57 (1.92%) cases had intracranial complications (IC) and 39 (1.35%) cases had extracranial complications (EC). They observed that meningitis and brain abscess were com-mon amongst the intracranial complications group, whereas subperi-osteal abscesses (mastoid and Bezolt&rsquo;s abscess) were common complications in the extracranial complications group. Despite advances in the development of antibiotics, we still have otogenic brain abscess in our country. Therefore, the aim of this study was to describe the clinical profile, treatment and surgical outcome of patients presenting with otogenic brain abscess.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Between January 1997 and January 2006, a total of 10,800 cases of chronic otitis media (COM) were followed-up in our clinic. Clinical records of patients in UKMMC with otogenic brain abscess were reviewed. The data analyzed included age, sex, clinical symptoms, site of abscess, treatment options and complications.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	There were 10 patients (2 females, 8 males) with a mean age of 42 (range 11 to 69) years. The mean follow-up period was 14 months. All patients had cholesteatoma. At the first presentation to the ENT Clinic, the patients were treated with a high index of suspicion and an urgent Computerized Tomography (CT) scan was performed. All patients had a history of chronic ear discharge, headache, otalgia and fever (Table I). Six of the 10 patients had cere-bellar abscesses (Figure 1) and 4 had temporal lobe abscesses (Figure 2). Cere-bellar signs were present in 6 patients. All the patients were admitted and treated and with broad-spectrum antibiotics. In 5 pa-tients, mastoid exploration was the primary surgical treatment and the brain abscesses were treated non-surgically. In the other 5 patients, craniotomy and drainage was performed followed by mastoid exploration when their neurological conditions had stabilized. One patient was admitted for 3 weeks for intravenous antibiotics as the brain abscess did not completely resolve after the repeat CT brain. There was no permanent neurological deficit in our pa-tients during follow up. There was also no mortality in this study. We advocate urgent CT scan of temporal bone and brain to be done and is mandatory in any patients presenting with ear discharge, moderate to severe headache, otalgia and fever.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	In this study we found cholesteatoma in all our patients with otogenic brain abscesses. They presented with a history of chronic ear discharge, headache, otalgia and fever.</p>\r\n<p>\r\n	The underlying pathology for cerebellar abscess was cholesteatoma. Cholesteato-ma produces enzymes that cause demine-ralization of bone. The infection can spread through this bony erosion into the posterior cranial fossa (PCF) and cause cerebellar abscess (Nadol and Schuknecht 1993).</p>\r\n<p>\r\n	The infection also can spread to the PCF through the internal auditory canal (IAC), which opens into it. Another possible route of spread is through inflammation of small veins (thrombophlebitis). Thrombophlebitis can spread in any direction and can cause temporal lobe abscess and other extra-cranial complications (i.e. mastoid abscess, perisinus abscess, subperiosteal abscess). According to Nadol and Schuknecht (1993)direct extension is the route commonly implicated in complications due to COM. Thrombophlebitis spread is more common in acute infections and in acute exacerba-tions of chronic infection (Nadol and Schuknecht 1993).</p>\r\n<p>\r\n	Cerebellar signs were present in all our patients with cerebellar abscess. Gait ataxia and nystagmus were the common-est complaints in this group. However, there were no other cerebellar signs such as dysarthria, dysdiadochokinesia, past-pointing and rebound phenomenon. We advocate that any COM patients with symptoms and signs of impending compli-cations of otitis media should be admitted urgently for parenteral antibiotic and further radiological investigations.</p>\r\n<p>\r\n	Clinical neurological examination is helpful in the localization of the abscess. Nominal aphasia may be associated with abscesses if located in the dominant temporal lobe. Other signs include quadrantic homonymous hemianopias (involving upper quadrants is more fre-quent than the lower quadrants) and con-tralateral limb weakness (Nadol and Schuknecht 1993).</p>\r\n<p>\r\n	All our patients had a history of fever. Glasscock and Shambaugh(1990) reported that fever in brain abscess, is usually low grade and some patients may have a subnormal temperature. Less than 50% of patients with brain abscess present with the classical triad of fever, headache and neurological deficit (Nadol and Schuknecht 1993). Youngs (1998) and Neely (1986) described signs and symp-toms of brain abscess according to the stages of its development. The first stage corresponds to the encephalitis stage that results from the invasion of brain tissue. The symptoms are general malaise, headache, fevers, chills, nausea and, vomiting. They are usually quite mild and often mimic an exacerbation of chronic otitis media.</p>\r\n<p>\r\n	During the second stage or &ldquo;latent stage&rdquo; the abscess localizes and the symptoms disappear. The third stage is characterized by signs and symptoms associated with both increased intracranial pressure and compression of specific structures in the brain. Severe headache is present in 50% to 60% of patients (Haines et al 1990). Nausea and vomiting (often projectile) occur in 25% to 50% of cases. About 20% to 30% of patients may present with seizures (Haines et al 1990). Sodden and Koch(2000) reported a child with an otogenic brain abscess presenting with febrile seizures. The third stage of brain abscess progression is when the abscess ruptures into the ventricle or the sub-arachnoid space. This will result in rapid clinical decline and ultimately death due to severe ventriculitis.</p>\r\n<p>\r\n	The factors that contribute to the spread of infection beyond the middle ear space depend on the species and virulence of the organism. Other factors are host immune system and prior therapy. Levent and Bulent (2000)reviewed 41 patients with otogenic brain abscess and reported localization in the temporal lobe in 54%, the cerebellum in 44% and in both locations in 2%. None of our patients had abscesses in both locations. They also found that all patients had COM and 95% had cholesteatoma. Jaran et al (1995) from Chiang Mai, Thailand reported 29 patients with otogenic brain abscess secondary to COM of whom 89.7% had cholesteatoma. Temporal lobe abscesses were seen in 20 (68.9%) and cerebellar abscesses in 9 patients(31.1%). Kurien et al(1998) reported that all their patients had cholesteatoma as in our group.</p>\r\n<p>\r\n	Laboratory investigations add little to the diagnosis of brain abscess. Britt (1985) reported that total white blood cell (TWBC) might be elevated or normal. CT with intravenous contrast is the imaging modality of choice in screening for complications of COM. It can demonstrate the site of complications, the extent of the abscess, and any bone involvement (Fitzpatrick and Gan 1999). Magnetic resonance imaging (MRI) with contrast and magnetic resonance angiography are useful in the definitive diagnosis of certain intracranial complications such as lateral sinus thrombosis. Both contrast enhanced CT or MRI will demonstrate ring enhance-ment for brain abscesses (Soden and Koch 2000, Marquadt et al 2000). The abscess is usually encircled by an area of low density representing oedema in the surrounding brain tissue (Fitzpatrick and Gan 1999).</p>\r\n<p>\r\n	Early treatment of brain abscess is mandatory. Management can be medical alone (intravenous antibiotics). This is usually reserved for patients with small abscesses (&lt; 3 cm in diameter), multiple lesions or those who are at high risk for surgery (Fitzpatrick and Gan 1999).&nbsp; Intravenous antibiotic therapy should be started as early as possible. Polymicrobial infection is common in COM and its complications (Nadol and Schucknecht 1993). Therefore&nbsp; broad-spectrum antibiotic coverage for aerobic and anaerobic organisms is recommended. Combination drug therapy may be necessary to accomplish this goal.</p>\r\n<p>\r\n	Surgical treatment may involve aspiration or excision of the abscess(Britt 1985). The treatment of the condition is tailored to the clinical presentation of each patient and neurological considerations. The import-ance of early and appropriate treatment of the abscess cannot be over-emphasized.</p>\r\n<p>\r\n	In general, however it is desirable to delay surgical treatment of the infected ear until the patient is neurologically and sys-temically stable. Clinical deterioration and progression of infection despite appropriate medical therapy are indications for earlier surgical intervention(Nadol and Schucknecht 1993). All our patients were treated with broad-spectrum antibiotics (3<sup>rd</sup> generation cephalosporins). In 50% of our patients mastoid exploration was the primary surgical treatment and the brain abscesses were treated conservatively. In the other 5 patients, craniotomy and drainage was performed followed by mastoid exploration when their neurological condition had stabilized. There was no mortality in this study. All our patients had an uneventful recovery. The mortality rates in other studies were 31% and 10% reported by Jaran et al (1995) and Levent and Bulent (2000) respectively.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The primary goal of management of otogenic brain abscess is to overcome the danger of brain abscess either by con-servative treatment with antibiotics or surgical drainage. Mastoid explorations are performed only when their neurological condition is stable. A high index of suspicion aided by contrast enhanced CT is important for early diagnosis and treatment.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors wish to express their appreciation to Mr. Kamaruzzaman for his assistance in photography.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(353, 350, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Primary&nbsp; adenocarcinoma&nbsp; of&nbsp; the fallopian tube&nbsp; is&nbsp; extremely&nbsp; rare&nbsp; and&nbsp; account&nbsp; for approximately&nbsp; 0.14-1.8%&nbsp; of&nbsp; all gynaeco- logical&nbsp; malignancies&nbsp; (Gadducci&nbsp; 2002). The classical triad of symptoms of pelvic pain,&nbsp; pelvic&nbsp; mass&nbsp; and a serosanguinous discharge, also known as &ldquo;hydrops tubae profluence&rdquo;&nbsp; described&nbsp; by&nbsp; Latzko&nbsp; in&nbsp; 1916 (Lawson&nbsp; et al.&nbsp; 1996),&nbsp; rarely&nbsp; helps&nbsp; to&nbsp; di- agnose such cases in clinical practice. In many reported cases tubal carcinoma are diagnosed&nbsp; during&nbsp; surgery&nbsp; for&nbsp; unrelated problems.&nbsp; We&nbsp; describe&nbsp; here&nbsp; a case&nbsp; of primary&nbsp; adenocarcinoma&nbsp; of&nbsp; the fallopian tube in a lady who presented with symptoms&nbsp; suggestive&nbsp; of&nbsp; pelvic&nbsp; inflammatory disease. The tumour was only confirmed following histopathological examination.</p>\r\n<p>\r\n	<strong>CASE REPORT&nbsp; </strong></p>\r\n<p>\r\n	A&nbsp; 50-year-old&nbsp; post-menopausal&nbsp; Indian lady (para 3) was referred from a periph- eral&nbsp; clinic&nbsp; for&nbsp; lower&nbsp; abdominal&nbsp; pain&nbsp; of three months&rsquo; duration. The pain that had worsened&nbsp; over&nbsp; the&nbsp; past&nbsp; two&nbsp; weeks&nbsp; was intermittent&nbsp; and&nbsp; pricking&nbsp; in&nbsp; nature&nbsp; and had&nbsp; radiated&nbsp; to&nbsp; the&nbsp; back.&nbsp; There&nbsp; was&nbsp; no history of fever or change in bowel or uri- nary&nbsp; habits.&nbsp; In&nbsp; addition,&nbsp; her&nbsp; symptoms were&nbsp; not&nbsp; associated&nbsp; with&nbsp; postmenopausal bleeding. The patient had been diag- nosed&nbsp; with&nbsp; diabetes&nbsp; mellitus,&nbsp; which&nbsp; was treated&nbsp; with&nbsp; oral&nbsp; antihyperglycaemics. She&nbsp; had&nbsp; a&nbsp; laparotomy&nbsp; 26&nbsp; years&nbsp; ago&nbsp; but the&nbsp; indications&nbsp; for&nbsp; the&nbsp; surgery&nbsp; and&nbsp; its findings&nbsp; were&nbsp; not&nbsp; apparent.&nbsp; She&nbsp; had&nbsp; bilateral&nbsp; tuboligation&nbsp; six&nbsp; years&nbsp; later.&nbsp;&nbsp;&nbsp; On physical&nbsp; examination&nbsp; the&nbsp; patient&nbsp; was comfortable&nbsp; and&nbsp; had&nbsp; no&nbsp; evidence&nbsp; of jaundice or pallor. Her abdomen was soft and no masses were felt. Interestingly, a pelvic&nbsp; examination&nbsp; did&nbsp; not&nbsp; reveal&nbsp; any mass in the adnexa or pouch of Douglas either.</p>\r\n<p>\r\n	An&nbsp; ultrasound&nbsp; scan&nbsp; revealed&nbsp; an empty uterus&nbsp; measuring&nbsp; 8.5x5.0&nbsp; cm&nbsp; with unremarkable&nbsp; endometrium.&nbsp; However,&nbsp; a tubular mass was noted on each side&nbsp; of the uterus, measuring 3.6x4.5 cm on the right&nbsp; and&nbsp; 5.4x2.7&nbsp; cm&nbsp; on the left.&nbsp; The ovaries were not enlarged and there was no&nbsp; ascites.&nbsp; A provisional&nbsp; diagnosis&nbsp; of pelvic inflammatory disease with bilateral hydrosalphinx was made.</p>\r\n<p>\r\n	Following&nbsp; this,&nbsp; a&nbsp; CT&nbsp; scan&nbsp; of&nbsp; the&nbsp; pelvis and&nbsp; abdomen&nbsp; was&nbsp; performed.&nbsp; It&nbsp; showed a&nbsp; bulky&nbsp; uterus&nbsp; with&nbsp; bilateral&nbsp; adnexal cysts. The left cyst measured 4.1x2.7 cm (Fig.1)&nbsp; and&nbsp; it&nbsp; contained&nbsp; a&nbsp; solid&nbsp; area measuring&nbsp; 1.5x1.9&nbsp; cm.&nbsp; The&nbsp; right&nbsp; cyst measured 3.1x1.6 cm with no solid areas within&nbsp; it.&nbsp; There&nbsp; was&nbsp; no&nbsp; evidence&nbsp; of ascites&nbsp; and&nbsp; the&nbsp; para-aortic&nbsp; lymph&nbsp; nodes were&nbsp; not&nbsp; enlarged.&nbsp; The&nbsp; liver&nbsp; showed&nbsp; a 2.2x1.5&nbsp; cm&nbsp; enhancing&nbsp; lesion&nbsp; in segment VI. A diagnosis of a malignant left ovarian cyst with liver metastasis was suggested.</p>\r\n<p>\r\n	At&nbsp; laparotomy,&nbsp; both&nbsp; fallopian&nbsp; tubes were&nbsp; dilated&nbsp; and&nbsp; elongated.&nbsp; The&nbsp; uterus, cervix and both ovaries were unremarka- ble. The Pouch of Douglas and omentum were normal and there was no ascites.&nbsp;&nbsp;</p>\r\n<p>\r\n	A&nbsp; total&nbsp; abdominal&nbsp; hysterectomy&nbsp; and bilateral&nbsp; salpingoophorectomy&nbsp; was&nbsp; performed.</p>\r\n<p>\r\n	Grossly,&nbsp; the&nbsp; left&nbsp; fallopian&nbsp; tube&nbsp; was markedly&nbsp; distended&nbsp; and&nbsp; measured&nbsp; 10x3 cm (Fig.2A). Cut section showed a mixed solid&nbsp; and&nbsp; cystic&nbsp; tumour&nbsp; within&nbsp; its&nbsp; lumen (Fig.2B).&nbsp; The&nbsp; right&nbsp; fallopian&nbsp; tube meas- ured&nbsp; 10x1.5&nbsp; cm. There&nbsp; was&nbsp; a small&nbsp; cyst (one&nbsp; cm)&nbsp; arising&nbsp; from&nbsp; the fimbrial&nbsp; end.&nbsp; Histological&nbsp; examination&nbsp; of&nbsp; the left&nbsp; fallo- pian&nbsp; tube&nbsp; tumour&nbsp; revealed&nbsp; the presence of a moderately differentiated, high grade adenocarcinoma&nbsp; forming&nbsp; papillary&nbsp; archi- tecture.&nbsp; The&nbsp; papillae&nbsp; forms&nbsp; irregular branching&nbsp; pattern&nbsp; with&nbsp; slit-like&nbsp; lumina (Fig.2C)&nbsp; and&nbsp; are&nbsp; highly&nbsp; cellular.&nbsp; The tumour&nbsp; infiltrated&nbsp; the&nbsp; muscular&nbsp; layer&nbsp; of the fallopian tube but it did not breach the serosa&nbsp; (Fig.2D).&nbsp; Mitosis&nbsp; was&nbsp; brisk (&gt;20/10hpf)&nbsp; (Fig.2E)&nbsp; and&nbsp; areas&nbsp; of necrosis&nbsp; were&nbsp; present.&nbsp; The&nbsp; uterus, cervix,&nbsp; both&nbsp; ovaries&nbsp; and&nbsp; right&nbsp; fallopian tube&nbsp; were&nbsp; free&nbsp; of&nbsp; tumour. Immunohistochemistry&nbsp; revealed&nbsp; diffuse expression&nbsp; of&nbsp; WT1&nbsp; and&nbsp; p53&nbsp; (Fig.2F)&nbsp; in the&nbsp; tumour&nbsp; cells.&nbsp; A&nbsp; diagnosis&nbsp; of&nbsp; high- grade serous adenocarcinoma of the left fallopian tube was made.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	Primary&nbsp; fallopian&nbsp; tube&nbsp; carcinoma&nbsp; is&nbsp; a rare&nbsp; malignancy&nbsp; of&nbsp; the&nbsp; female&nbsp; genital tract&nbsp; that&nbsp; is&nbsp; rarely&nbsp; diagnosed&nbsp; preopera- tively. The tumour is often mistaken for a benign&nbsp; pelvic&nbsp; disease&nbsp; or&nbsp; ovarian&nbsp; cancer. However,&nbsp; in&nbsp; contrast&nbsp; to&nbsp; ovarian&nbsp; carci- noma,&nbsp; fallopian&nbsp; tube&nbsp; malignancies&nbsp; more often&nbsp; present&nbsp; at&nbsp; an&nbsp; early&nbsp; stage&nbsp; but&nbsp; are usually&nbsp; high&nbsp; grade&nbsp; with&nbsp; a&nbsp; worse&nbsp; progno- sis,&nbsp; stage&nbsp; for&nbsp; stage.&nbsp; The&nbsp; tumour&nbsp; usually originates in the ampulla and histological pattern&nbsp; reproduces&nbsp; the&nbsp; epithelium&nbsp; of&nbsp; the fallopian&nbsp; tube&nbsp; mucosa,&nbsp; frequently&nbsp; pre- senting&nbsp; as&nbsp; serous&nbsp; adenocarcinoma (Salvador et al. 2009).&nbsp; &nbsp;&nbsp;</p>\r\n<p>\r\n	In&nbsp; the&nbsp; present&nbsp; case,&nbsp; the&nbsp; patient&nbsp; pre- sented&nbsp; with&nbsp; symptoms&nbsp; of&nbsp; pelvic&nbsp; inflam- matory&nbsp; disease&nbsp; that&nbsp; failed&nbsp; to&nbsp; respond&nbsp; to antibiotic&nbsp; treatment.&nbsp; A&nbsp; decision&nbsp; to&nbsp; pro- ceed&nbsp; with a laparotomy&nbsp; was made when the&nbsp; CT&nbsp; scan&nbsp; reported&nbsp; a&nbsp; possible&nbsp; malig- nant&nbsp; ovarian&nbsp; tumour.&nbsp; In&nbsp; retrospect,&nbsp; an intraoperative&nbsp; frozen&nbsp; section&nbsp; of&nbsp; the&nbsp; fallo- pian tube would have helped decide on a more definitive surgical procedure.</p>\r\n<p>\r\n	The&nbsp; presentation&nbsp; of&nbsp; fallopian&nbsp; tube&nbsp; car- cinoma&nbsp; as&nbsp; pelvic&nbsp; inflammatory&nbsp; disease has&nbsp; also&nbsp; been&nbsp; reported&nbsp; by&nbsp; others (Cristalli&nbsp; et&nbsp; al.&nbsp; 1992,&nbsp; Kurjak&nbsp; et&nbsp; al.&nbsp; 1998). In&nbsp; addition,&nbsp; the&nbsp; tumour&nbsp; may&nbsp; mimic&nbsp; tubo- ovarian abscess that failed to respond to therapy&nbsp; as&nbsp; reported&nbsp; by&nbsp; Halperin&nbsp; et&nbsp; al. (2005).&nbsp; In&nbsp; their&nbsp; report,&nbsp; malignant&nbsp; cells were&nbsp; aspirated&nbsp; from&nbsp; sonographically guided&nbsp; drainage&nbsp; of&nbsp; the&nbsp; suspected&nbsp; tubo- ovarian&nbsp; abscess&nbsp; and&nbsp; the&nbsp; CA125&nbsp; levels were&nbsp; elevated&nbsp; prior&nbsp; to&nbsp; surgery,&nbsp; further supporting&nbsp; a&nbsp; malignant&nbsp; pathology.&nbsp; Previ- ous&nbsp; reports&nbsp; also&nbsp; emphasized&nbsp; the&nbsp; poorer prognosis fallopian tube adenocarcinoma carry&nbsp; compared&nbsp; to&nbsp; ovarian&nbsp; carcinoma. Five-year survival of patients with stage I disease is only about 60% and only 10% of patients with advanced disease will be cured (Ng and Lawton, 1998). In conclu- sion, fallopian&nbsp; tube carcinoma should&nbsp; be considered&nbsp; as&nbsp; a&nbsp; possible&nbsp; diagnosis&nbsp; in females&nbsp; diagnosed&nbsp; with&nbsp; pelvic&nbsp; inflam- matory&nbsp; disease&nbsp; but&nbsp; do&nbsp; not&nbsp; show&nbsp; signifi- cant respond to treatment.</p>\r\n', 1),
(360, 357, '<p class="rtejustify">\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p class="rtejustify">\r\n	Acute&nbsp; lymphoblastic&nbsp; leukaemia&nbsp; (ALL)&nbsp; is one&nbsp; of&nbsp; the&nbsp; common&nbsp; malignancies&nbsp; of&nbsp; the pediatric age group. It results from clonal proliferation&nbsp; of&nbsp; lymphoid&nbsp; precursors&nbsp; with arrested&nbsp; maturation.&nbsp; The&nbsp; disease&nbsp; can originate&nbsp; from&nbsp; lymphoid&nbsp; cells&nbsp; of&nbsp; B-&nbsp; or&nbsp; T- lineage,&nbsp; although&nbsp; mixed&nbsp; lineage&nbsp; leukae- mias&nbsp; do&nbsp; rarely&nbsp; occur.&nbsp; Its&nbsp; classification&nbsp; is based&nbsp; on&nbsp; morphological&nbsp; and&nbsp; cytochemi- cal&nbsp; characteristics&nbsp; established&nbsp; by&nbsp; the French-American-British&nbsp; (FAB)&nbsp; group (Bennett et al. 1981). Today, the diagno-sis&nbsp; of&nbsp; ALL&nbsp; is&nbsp; further&nbsp; facilitated&nbsp; by&nbsp; flow cytometry.&nbsp; Flow&nbsp; cytometric&nbsp; analysis&nbsp; al- lows&nbsp; for&nbsp; sub-classification&nbsp; of&nbsp; ALL&nbsp; by&nbsp; uti- lizing&nbsp; flourochrome-labelled&nbsp; monoclonal antibodies.&nbsp; About&nbsp; 60%&nbsp; of&nbsp; ALL&nbsp; patients have early pre-B immunophenotype. The blast&nbsp; cells&nbsp; usually&nbsp; express&nbsp; CD&nbsp; 19, cytoplasmic&nbsp; CD&nbsp; 22,&nbsp; CD&nbsp; 79&alpha;,&nbsp; CD&nbsp; 10, terminal&nbsp; deoxynucleotidyl&nbsp; transferase (TdT)&nbsp; and&nbsp; 75%&nbsp; express&nbsp; CD&nbsp; 34.&nbsp; T-cell ALL&nbsp; accounts&nbsp; for&nbsp; 12-15%&nbsp; of&nbsp; ALL&nbsp; cases. The&nbsp; blast&nbsp; cells&nbsp; express&nbsp; surface&nbsp; CD&nbsp; 7, cytoplasmic CD 3, CD 2, CD 5 and TdT.&nbsp; The&nbsp; blast&nbsp; cells&nbsp; may&nbsp; also&nbsp; express aberrant antigens which do not belong to the&nbsp; main&nbsp; lineage.&nbsp;&nbsp;&nbsp; Immunological characterization&nbsp; of&nbsp; acute&nbsp; leukaemia&nbsp; is therefore essential in defining cell lineage to&nbsp; aid&nbsp; treatment&nbsp; options&nbsp; and determination of prognosis. The&nbsp; lineage&nbsp; of&nbsp; the leukaemic&nbsp; blast usually&nbsp; remains&nbsp; similar&nbsp; throughout&nbsp; the disease&nbsp; course,&nbsp; although&nbsp; the pattern&nbsp; of antigen expressions may change slightly.&nbsp; However,&nbsp; a switch&nbsp; of&nbsp; lineage&nbsp; of&nbsp; blast cells&nbsp; does&nbsp; rarely&nbsp; occur.&nbsp; Pui et al. described&nbsp; cases&nbsp; which&nbsp; showed&nbsp; switch from&nbsp; lymphoid&nbsp; to myeloid&nbsp; phenotype&nbsp; or the&nbsp; reverse&nbsp; (Pui&nbsp; et&nbsp; al.&nbsp; 1986).&nbsp; From literature&nbsp; review,&nbsp; phenotypic&nbsp; lineage switch rarely occurs from T-ALL to B-ALL or&nbsp; vice&nbsp; versa&nbsp; and&nbsp; to&nbsp; the&nbsp; best&nbsp; of&nbsp; our knowledge, only one case has described this&nbsp; phenomenon&nbsp; (Besalduch&nbsp; et&nbsp; al. 1990).&nbsp; Our&nbsp; case&nbsp; illustrates&nbsp; an unusual phenotypic switch in leukaemic cells from a&nbsp; patient&nbsp; who first&nbsp; presented&nbsp; with&nbsp; T-cell acute&nbsp; lymphoblastic&nbsp; leukaemia&nbsp; who&nbsp; had gone&nbsp; into&nbsp; remission&nbsp; and&nbsp; relapsed&nbsp; seven months later with common pre-B ALL.&nbsp;</p>\r\n<p class="rtejustify">\r\n	<strong>CASE REPORT </strong></p>\r\n<p class="rtejustify">\r\n	A&nbsp; 10-year-old&nbsp; boy&nbsp; presented&nbsp; with&nbsp; two weeks history of low grade fever, lethargy and&nbsp; poor&nbsp; appetite.&nbsp; The&nbsp; patient&nbsp; was&nbsp; the youngest&nbsp; of&nbsp; four&nbsp; siblings.&nbsp; His&nbsp; father,&nbsp; a 36-year-old&nbsp; man&nbsp; worked&nbsp; as&nbsp; a printing officer&nbsp; at a private&nbsp; company&nbsp; and his mother,&nbsp; a 36-year-old&nbsp; woman,&nbsp; was&nbsp; a housewife.</p>\r\n<p class="rtejustify">\r\n	At&nbsp; presentation,&nbsp; the&nbsp; patient&nbsp; was&nbsp; pale and&nbsp; in&nbsp; respiratory&nbsp; distress.&nbsp; His&nbsp; blood pressure&nbsp; was&nbsp; 105/40&nbsp; mmHg,&nbsp; pulse&nbsp; rate of&nbsp; 168 bpm&nbsp; and oxygen&nbsp; saturation&nbsp; was 100%&nbsp; under&nbsp; nasal&nbsp; prong&nbsp; oxygen.&nbsp; Physi- cal examination revealed pleural effusion over&nbsp; the&nbsp; right&nbsp; lung&nbsp; and cardiomegaly.&nbsp; There&nbsp; were&nbsp; hepatosplenomegaly&nbsp; mea- suring 5 cm and 4 cm below the subcos- tal margins respectively.</p>\r\n<p class="rtejustify">\r\n	Laboratory investigation results showed haemoglobin&nbsp; (Hb)&nbsp; 4.9&nbsp; g/dL,&nbsp; total&nbsp; white blood&nbsp; cells&nbsp; (WBC)&nbsp; 41.5x109/l,&nbsp; platelets 37x109/l,&nbsp; neutrophils&nbsp; 0.8x109/l,&nbsp; lym- phocytes&nbsp; 38.6x109/l.&nbsp; The&nbsp; peripheral blood smear showed presence of numer- ous&nbsp; blast&nbsp; cells.&nbsp; The&nbsp; blast&nbsp; cells&nbsp; exhibited high&nbsp; nuclear&nbsp; to&nbsp; cytoplasmic&nbsp; ratio&nbsp; with&nbsp; in- conspicuous nucleoli (Figure 1).</p>\r\n<p class="rtejustify">\r\n	His&nbsp; chest&nbsp; radiograph&nbsp; showed&nbsp; cardi- omegaly&nbsp; with&nbsp; mediastinal&nbsp; widening.&nbsp; The bone&nbsp; marrow&nbsp; aspirate&nbsp; showed&nbsp; presence of more than 90% blast cells which were homogenous&nbsp; in&nbsp; size,&nbsp; showing&nbsp; high&nbsp; nuc- lear&nbsp; to&nbsp; cytoplasmic&nbsp; ratio&nbsp; with&nbsp; basophilic cytoplasm&nbsp; and&nbsp; inconspicuous&nbsp; nucleoli. Other&nbsp; cell&nbsp; lines&nbsp; were&nbsp; depressed.&nbsp; The blast&nbsp; cells&nbsp; were&nbsp; negative&nbsp; for&nbsp; peroxidase and&nbsp; periodic&nbsp; acid&nbsp; Schiff&nbsp; stains.&nbsp; These findings&nbsp; were&nbsp; consistent&nbsp; with&nbsp; acute&nbsp; lym- phoblastic&nbsp; leukaemia,&nbsp; FAB-L1.&nbsp; The&nbsp; tre- phine&nbsp; biopsy&nbsp; showed&nbsp; similar&nbsp; findings.&nbsp; Flow cytometric analysis of the peripheral blood and bone marrow aspirate showed that the population was positive for T-cell markers; CD 2, intracellular CD 3, CD 4, CD 5, CD 7, CD 8, HLA-DR but negative for CD 19, CD 79a, CD 56, CD 117, intra MPO, CD 13 and CD 33 (Figure 2). Marrow&nbsp; cytogenetic&nbsp; study&nbsp; revealed&nbsp; a 46 XY normal karyotype with negative BCR-ABL translocation.</p>\r\n<p class="rtejustify">\r\n	The patient achieved remission with UK ALL 97 protocol regime B induction che- motherapy.&nbsp; However,&nbsp; at&nbsp; week&nbsp; 20,&nbsp; his bone marrow aspirate revealed presence of&nbsp; excess&nbsp; blast&nbsp; cells&nbsp; of&nbsp; 10%.&nbsp; Flow&nbsp; cyto- metry&nbsp; analysis&nbsp; was&nbsp; inconclusive&nbsp; due&nbsp; to sample&nbsp; inadequacy.&nbsp; He&nbsp; subsequently received&nbsp; Doxorubicin,&nbsp; Vincristine,&nbsp; Dex- amethasone&nbsp; and Asparaginase&nbsp; as&nbsp; per delayed&nbsp; intensification&nbsp; 1 chemotherapy protocol which was complicated by bron- chopneumonia&nbsp; secondary&nbsp; to febrile&nbsp; neu- tropaenia.&nbsp; The&nbsp; repeat&nbsp; bone&nbsp; marrow&nbsp; ex- amination&nbsp; at&nbsp; week&nbsp; 25 showed&nbsp; persis- tence&nbsp; of&nbsp; excess&nbsp; of&nbsp; blast&nbsp; cells&nbsp; (7%).&nbsp; The bone&nbsp; marrow&nbsp; examination&nbsp; was&nbsp; repeated at&nbsp; week&nbsp; 29 and&nbsp; showed&nbsp; presence&nbsp; of more&nbsp; than 26%&nbsp; blast&nbsp; cells&nbsp; which&nbsp; com- prised&nbsp; of&nbsp; two&nbsp; populations&nbsp; of&nbsp; blast&nbsp; cells (Figure&nbsp; 3).&nbsp; One&nbsp; population&nbsp; was&nbsp; larger with&nbsp; abundant&nbsp; basophilic&nbsp; cytoplasm (morphologically&nbsp; L2 blast cells)&nbsp; while the other&nbsp; population&nbsp; was&nbsp; smaller,&nbsp; more&nbsp; ho- mogenous&nbsp; with&nbsp; very&nbsp; scanty&nbsp; cytoplasm (L1&nbsp; blast&nbsp; cells).&nbsp; The&nbsp; two&nbsp; populations&nbsp; of blast&nbsp; cells&nbsp; showed&nbsp; variable&nbsp; antigen&nbsp; ex- pressions. Both populations of blast cells expressed&nbsp; CD&nbsp; 19,&nbsp; anti-HLA-DR,&nbsp; CD&nbsp; 10 and&nbsp; cytoplasmic&nbsp; CD&nbsp; 22. One&nbsp; population of&nbsp; blast&nbsp; cells&nbsp; was&nbsp; CD&nbsp; 34&nbsp; positive&nbsp; with negative&nbsp; CD&nbsp; 20 while&nbsp; the&nbsp; other&nbsp; popula-tion&nbsp; was&nbsp; CD&nbsp; 34 negative&nbsp; with&nbsp; positive cytoplasmic&nbsp; CD&nbsp; 20. The&nbsp; blast&nbsp; cells&nbsp; were negative&nbsp; for&nbsp; intra&nbsp; CD&nbsp; 3, CD&nbsp; 7, CD&nbsp; 117, intra-MPO, CD 13 and CD 33 (Figure 4). A&nbsp; diagnosis&nbsp; of&nbsp; precursor&nbsp; B-cell&nbsp; acute lymphoblastic&nbsp; leukaemia&nbsp; with&nbsp; CD&nbsp; 10 positivity&nbsp; (CALLA)&nbsp; was&nbsp; then&nbsp; made&nbsp; and the&nbsp; patient&nbsp; was&nbsp; started&nbsp; on&nbsp; the German Relapse&nbsp; Protocol&nbsp; (GRP).&nbsp; Repeat&nbsp; bone marrow examination after four months of relapse&nbsp; showed&nbsp; excess&nbsp; of&nbsp; blast&nbsp; cells (10%).&nbsp; The&nbsp; blast&nbsp; cells&nbsp; were mostly&nbsp; large in&nbsp; size&nbsp; with&nbsp; relatively&nbsp; abundant&nbsp; cytop- lasm.&nbsp; A second&nbsp; course&nbsp; of chemotherapy was&nbsp; given&nbsp; for&nbsp; eight&nbsp; days.</p>\r\n<p class="rtejustify">\r\n	Review&nbsp; of&nbsp; bone&nbsp; marrow&nbsp; examination after&nbsp; completing&nbsp; two&nbsp; courses&nbsp; of&nbsp; chemo- therapy showed persistence of blast cells (10%&nbsp; blast&nbsp; cells).&nbsp;&nbsp;&nbsp; In&nbsp; view&nbsp; of&nbsp; this&nbsp; condi- tion,&nbsp; bone&nbsp; marrow&nbsp; transplant&nbsp; was&nbsp; no longer&nbsp; a&nbsp; therapeutic&nbsp; option.&nbsp; The&nbsp; man- agement&nbsp; was&nbsp; then&nbsp; changed&nbsp; to&nbsp; palliative care which included low-dose chemothe- rapy and prophylactic treatment for infec- tions.</p>\r\n<p class="rtejustify">\r\n	<strong>DISCUSSION&nbsp; </strong></p>\r\n<p class="rtejustify">\r\n	Leukaemic stem cells have heterogenous differentiation potential.&nbsp; It has been well described&nbsp; in&nbsp; various&nbsp; articles&nbsp; that&nbsp; many different&nbsp; patterns&nbsp; of&nbsp; inappropriate&nbsp; ex- pression&nbsp; of&nbsp; lineage&nbsp; markers&nbsp; were&nbsp; ob- served&nbsp; in&nbsp; acute&nbsp; leukaemias.&nbsp; The&nbsp; spec- trum of mixed acute leukaemias includes biphenotypic&nbsp; leukaemia&nbsp; with&nbsp; markers&nbsp; of more than one lineage of antigen expres- sions&nbsp; present&nbsp; on&nbsp; the&nbsp; same&nbsp; blast&nbsp; cells, bilineal&nbsp; leukaemia&nbsp; with&nbsp; two&nbsp; distinct&nbsp; pop- ulations, and biclonal leukaemia with two distinct&nbsp; leukaemic&nbsp; cells&nbsp; of&nbsp; independent origins&nbsp; (Hoffbrand&nbsp; et&nbsp; al.&nbsp; 1988).&nbsp; These terms&nbsp; have&nbsp; recently&nbsp; however&nbsp; been&nbsp; re- vised&nbsp; in&nbsp; the&nbsp; WHO&nbsp; classification&nbsp; 2008.&nbsp; The&nbsp; term&nbsp; mixed&nbsp; phenotypic&nbsp; acute&nbsp; leu- kaemia&nbsp; (MPAL)&nbsp; is&nbsp; now&nbsp; applied&nbsp; to&nbsp; both biphenotypic&nbsp; and&nbsp; bilineal&nbsp; leukaemia&nbsp; in general,&nbsp; with&nbsp; more&nbsp; specific&nbsp; terms B/myeloid&nbsp; (B/MY)&nbsp; and&nbsp; T/myeloid&nbsp; (T/MY) leukaemia&nbsp; which&nbsp; refer&nbsp; to&nbsp; leukaemias containing&nbsp; the&nbsp; lineages&nbsp; specified,&nbsp; irres- pective of whether one or more than one population of blast cells is seen (Borowitz et&nbsp; al.&nbsp; 2008).&nbsp; There&nbsp; are&nbsp; cases&nbsp; of&nbsp; MPAL based&nbsp; on&nbsp; one&nbsp; criterion&nbsp; at&nbsp; diagnosis&nbsp; (e.g biphenotypic&nbsp; leukaemia)&nbsp; which&nbsp; change over time at relapse to the other (bilineal leukaemia),&nbsp; or&nbsp; vice&nbsp; versa.&nbsp; Furthermore, following&nbsp; therapy,&nbsp; persistent&nbsp; disease&nbsp; or relapse may occur as either pure ALL or AML.&nbsp; In&nbsp; some&nbsp; cases,&nbsp; this&nbsp; phenomenon has been termed &lsquo;lineage switch&rsquo;.&nbsp;</p>\r\n<p class="rtejustify">\r\n	The&nbsp; phenotypes&nbsp; of&nbsp; blast&nbsp; cells&nbsp; from&nbsp; pa- tients&nbsp; with&nbsp; ALL&nbsp; upon&nbsp; relapse&nbsp; most&nbsp; often adhere to the original lineage (Greaves et al. 1980). However, blast cells which un- dergo a &lsquo;lineage switch&rsquo; during the course of&nbsp; disease&nbsp; especially&nbsp; during&nbsp; relapse&nbsp; had been&nbsp; estimated&nbsp; in&nbsp; some&nbsp; series&nbsp; to&nbsp; be&nbsp; as high&nbsp; as&nbsp; 8%&nbsp; (Stass&nbsp; et&nbsp; al.&nbsp; 1986).&nbsp; In&nbsp; this case, a phenotypic lineage switch is seen where&nbsp; the&nbsp; blast&nbsp; cells&nbsp; initially&nbsp; expressed T-cell&nbsp; markers&nbsp; (cytoCD3+,&nbsp; CD2+,&nbsp; CD4+, CD8+,&nbsp; CD5+,&nbsp; CD7+)&nbsp; at&nbsp; diagnosis.&nbsp; How- ever at relapse, the blast cells were posi- tive&nbsp; for&nbsp; B-cell&nbsp; markers&nbsp; (CD19+,&nbsp; in- traCD22&nbsp; and&nbsp; CD20)&nbsp; and&nbsp; also&nbsp; showed positivity&nbsp; towards&nbsp; CD10,&nbsp; CD34,&nbsp; HLA-DR but&nbsp; negative&nbsp; for&nbsp; cytoplasmic&nbsp; CD3,&nbsp; CD7, CD117 and anti-MPO.</p>\r\n<p class="rtejustify">\r\n	The&nbsp; mechanism&nbsp; of lineage&nbsp; switch&nbsp; re- mains unclear. Most cases demonstrated consistent&nbsp; chromosomal&nbsp; findings&nbsp; at&nbsp; the time&nbsp; of&nbsp; diagnosis&nbsp; and switch&nbsp; and were therefore&nbsp; thought&nbsp; to be&nbsp; clonal&nbsp; in origin (Stass&nbsp; et al.&nbsp; 1984,&nbsp; Mantadakis&nbsp; et al. 2007).&nbsp; It&nbsp; was&nbsp; hypothesized&nbsp; that chemo- therapy&nbsp; might&nbsp; have&nbsp; induced&nbsp; suppression of a clone apparent at diagnosis, allowing the&nbsp; expansion&nbsp; of&nbsp; another&nbsp; clone&nbsp; with&nbsp; dif- ferent&nbsp; markers,&nbsp; which&nbsp; was&nbsp; resistant&nbsp; to treatment&nbsp; (Stass&nbsp; et al.&nbsp; 1984).&nbsp; Alterna- tively,&nbsp; the&nbsp; therapy&nbsp; might&nbsp; have&nbsp; a leuke- mogenic&nbsp; effect&nbsp; on a normal&nbsp; hemopoietic cell&nbsp; and&nbsp; the&nbsp; lineage&nbsp; switch&nbsp; might&nbsp; then represent&nbsp; the&nbsp; induction&nbsp; of&nbsp; a new&nbsp; malig- nancy&nbsp; rather&nbsp; than&nbsp; transformation&nbsp; of&nbsp; the initial&nbsp; leukaemic&nbsp; cells.&nbsp; In support&nbsp; of&nbsp; this view,&nbsp; there&nbsp; were&nbsp; cases&nbsp; described&nbsp; with&nbsp; a complete&nbsp; variation&nbsp; of&nbsp; cytogenetic&nbsp; mark- ers at diagnosis and relapse (Park et al. 2011)&nbsp; and&nbsp; after&nbsp; allogenic&nbsp; bone marrow transplantation&nbsp; (Brito-Babapulle&nbsp; et&nbsp; al. 1989).&nbsp; However,&nbsp; therapy-induced&nbsp; acute leukaemia&nbsp; had a long&nbsp; latency&nbsp; period&nbsp; of about 69 months as described in a large multicentre&nbsp; GIMEMA&nbsp; trial&nbsp; reported&nbsp; by Pagano et al. in 1998.</p>\r\n<p class="rtejustify">\r\n	As&nbsp; in this&nbsp; case,&nbsp; there&nbsp; was&nbsp; a rapid&nbsp; li- neage&nbsp; switch&nbsp; with&nbsp; an interval&nbsp; of&nbsp; just seven&nbsp; months&nbsp; from&nbsp; first&nbsp; presentation&nbsp; to relapse. As the time frame was too short, it&nbsp; was&nbsp; very&nbsp; unlikely&nbsp; for&nbsp; secondary&nbsp; malig- nancy&nbsp; to have&nbsp; set&nbsp; in.&nbsp; Thus,&nbsp; the most likely pathogenesis for the lineage switch in&nbsp; this&nbsp; case&nbsp; was&nbsp; chemotherapy-induced suppression&nbsp; of&nbsp; the apparent&nbsp; leukaemic clone at diagnosis, while allowing the ex- pansion&nbsp; of&nbsp; another&nbsp; clone&nbsp; with&nbsp; different markers&nbsp; which&nbsp; was&nbsp; resistant&nbsp; to treat- ment.&nbsp; A similar&nbsp; conclusion&nbsp; was&nbsp; also reached&nbsp; by&nbsp; Shivarov&nbsp; et al.&nbsp; who&nbsp; reported a very early onset lineage switch from B- ALL&nbsp; to AML in an adult&nbsp; patient&nbsp; within&nbsp; a period&nbsp; of&nbsp; only&nbsp; 3 months&nbsp; after&nbsp; the initial diagnosis (Shivarov et al. 2009).</p>\r\n<p class="rtejustify">\r\n	A&nbsp; variety&nbsp; of&nbsp; genetic&nbsp; anomalies&nbsp; has been&nbsp; reported&nbsp; in cases&nbsp; of&nbsp; ambiguous lineage&nbsp; leukaemias&nbsp; especially&nbsp; mixed phenotype&nbsp; acute&nbsp; leukaemia.&nbsp; The t(9;22)(q34;q11) BCR-ABL1 translocation and&nbsp; translocations&nbsp; associated&nbsp; with&nbsp; the MLL gene are the two most common ge- netic&nbsp; anomalies&nbsp; associated&nbsp; with&nbsp; MPAL.</p>\r\n<p class="rtejustify">\r\n	In&nbsp; the case&nbsp; of&nbsp; lineage&nbsp; switch,&nbsp; the&nbsp; data are scanty, but two cases have been re- ported&nbsp; to&nbsp; involve&nbsp; t (9;22)(q34;q11)&nbsp; BCR- ABL1&nbsp; translocation&nbsp; in two&nbsp; cases&nbsp; of Ph+ ALL to Ph+AML (Pane et al. 1996, Rear- don et al. 1994).</p>\r\n<p class="rtejustify">\r\n	In&nbsp; a&nbsp; study&nbsp; of&nbsp; immunophenotypic&nbsp; and cytogenetic changes at relapse by Hur et al.&nbsp; in&nbsp; 2001,&nbsp; of&nbsp;&nbsp;&nbsp; 99&nbsp; patients,&nbsp; 51&nbsp; patients (51.5%) had immunophenotypic changes at&nbsp; relapse&nbsp; with&nbsp; expression&nbsp; of&nbsp; aberrant markers&nbsp; to&nbsp; be&nbsp; more&nbsp; frequent&nbsp; at&nbsp; relapse than&nbsp; at&nbsp; initial&nbsp; diagnosis&nbsp; especially&nbsp; in&nbsp; B lineage&nbsp; ALL&nbsp; (41.1%&nbsp; versus&nbsp; 10.3%).&nbsp; Cy- togenetic&nbsp; changes&nbsp; at&nbsp; relapse&nbsp; were&nbsp; ob- served in 28 of 46 patients (60.8%). The initial&nbsp; abnormal&nbsp; karyotypes&nbsp; were&nbsp; more frequently&nbsp; associated&nbsp; with&nbsp; clonal changes&nbsp; at&nbsp; relapse&nbsp; compared&nbsp; to&nbsp; initially normal&nbsp; karyotypes&nbsp; (78%&nbsp; versus&nbsp; 43%) (Hur et al. 2001).&nbsp;</p>\r\n<p class="rtejustify">\r\n	Loss of CD10 and other antigens at re- lapse has been reported in about 15% of ALL&nbsp; patients,&nbsp; however&nbsp; a&nbsp; complete change&nbsp; of&nbsp; phenotype&nbsp; from&nbsp; T-cell&nbsp; ALL&nbsp; to B-cell ALL is rare. From literature search, only&nbsp; one&nbsp; case&nbsp; in&nbsp; 1990&nbsp; had&nbsp; the&nbsp; same phenomenon.&nbsp; Besalduch&nbsp; et&nbsp; al.&nbsp; in&nbsp; 1990 described&nbsp; a&nbsp; case&nbsp; of&nbsp; a&nbsp; 17-year&nbsp; old&nbsp; boy who&nbsp; first&nbsp; presented&nbsp; with&nbsp; massive&nbsp; hepa- tosplenomegaly&nbsp; and&nbsp; a&nbsp; mediastinal&nbsp; mass and&nbsp; was&nbsp; diagnosed&nbsp; to&nbsp; have&nbsp; T-cell&nbsp; acute lymphoblastic&nbsp; leukaemia.&nbsp; He&nbsp; was&nbsp; given standard&nbsp; chemotherapy&nbsp; and&nbsp; achieved remission. However, he relapsed 4 years later as precursor B-ALL.&nbsp;</p>\r\n<p class="rtejustify">\r\n	In a study by Stass et al. in a series of 264&nbsp; newly&nbsp; diagnosed&nbsp;&nbsp;&nbsp; acute&nbsp; leukaemia, of&nbsp;&nbsp;&nbsp; the&nbsp; 239&nbsp; patients&nbsp; who&nbsp; attained&nbsp; com- plete&nbsp; remission,&nbsp; 89&nbsp; relapsed&nbsp; with&nbsp; six cases&nbsp; (6.7%)&nbsp; demonstrating&nbsp; lineage switch&nbsp; from&nbsp; acute&nbsp; lymphoblastic&nbsp; leukae- mia&nbsp; to&nbsp; acute&nbsp; myeloid&nbsp; leukaemia&nbsp; or&nbsp; vice versa.&nbsp; Of&nbsp; the&nbsp; six&nbsp; cases,&nbsp; four&nbsp; had&nbsp; T-cell associated&nbsp; characteristics&nbsp; either&nbsp; at&nbsp; diag- nosis (three cases) or at lineage conver- sion&nbsp; (one&nbsp; case)&nbsp; suggesting&nbsp; that&nbsp; T&nbsp; lymphoblasts&nbsp; may&nbsp; have&nbsp; the&nbsp; potential&nbsp; for myeloid&nbsp; differentiation&nbsp; or&nbsp; that&nbsp; there&nbsp; may be&nbsp; a&nbsp; multipotential&nbsp; progenitor&nbsp; cell&nbsp; capa- ble of both T lymphoblast and myeloblas- tic&nbsp; differentiation.&nbsp; Of&nbsp; 30&nbsp; patients&nbsp; with&nbsp; T- ALL&nbsp; at&nbsp; diagnosis,&nbsp; 10&nbsp; of&nbsp; the&nbsp; 89&nbsp; patients who relapsed had T-cell ALL. Thus, 30% of&nbsp; T-ALL&nbsp; patients&nbsp; who&nbsp; relapsed&nbsp; demon- strated&nbsp; lineage&nbsp; switch.&nbsp; The&nbsp; issue&nbsp; is&nbsp; hig- hlighted&nbsp; here&nbsp; to&nbsp; show&nbsp; the&nbsp; heterogeneity of&nbsp; T-lymphoblast&nbsp; differentiation&nbsp; which,&nbsp; in this&nbsp; case&nbsp; evolved&nbsp; to&nbsp; B-lymphoblast&nbsp; at lineage&nbsp; conversion.&nbsp; This&nbsp; study&nbsp; also&nbsp; indi- cated&nbsp; that&nbsp; lineage&nbsp; switch&nbsp; in&nbsp; acute&nbsp; leu- kaemia&nbsp; was&nbsp; more&nbsp; common&nbsp; than&nbsp; might have been expected, possibly because of the wider use of intensive chemotherapy. More studies are needed to identify the target&nbsp; cells&nbsp; in&nbsp; leukaemogenesis&nbsp; to&nbsp; eva- luate&nbsp; their&nbsp; spontaneous&nbsp; or&nbsp; drug&nbsp; induced capacity&nbsp; for&nbsp; lineage&nbsp; deviation&nbsp; and&nbsp; diffe- rentiation,&nbsp; and&nbsp; to&nbsp; elucidate&nbsp; the&nbsp; relation- ship between the genetic and phenotypic characteristics of leukaemic cell lineages. Prompt&nbsp; recognition&nbsp; of&nbsp; lineage&nbsp; switch may&nbsp; be&nbsp; helpful&nbsp; in&nbsp; selecting&nbsp; an&nbsp; effective therapeutic&nbsp; regimen.&nbsp; Ultimately,&nbsp; therapy appropriate&nbsp; for&nbsp; the&nbsp; leukaemic&nbsp; phenotype at the time of lineage switch may be ad- visable.&nbsp;</p>\r\n<p class="rtejustify">\r\n	<strong>CONCLUSION </strong></p>\r\n<p class="rtejustify">\r\n	Conversion&nbsp; of&nbsp; T-cell&nbsp; lineage&nbsp; ALL&nbsp; to&nbsp; B-cell lineage ALL is a rare occurrence. This case illustrated&nbsp; a phenotypic&nbsp; lineage switch from a characteristic T-ALL to common precursor B-ALL.&nbsp; In&nbsp; our&nbsp; case,&nbsp; lineage&nbsp; conversion occurred&nbsp; seven&nbsp; months&nbsp; after&nbsp; the&nbsp; initial diagnosis which was postulated to be likely due&nbsp; to&nbsp; chemotherapy&nbsp; which&nbsp; eradicated&nbsp; the dominant&nbsp; clone&nbsp; apparent&nbsp; at&nbsp; diagnosis, permitting&nbsp; expansion&nbsp; of&nbsp; a&nbsp; secondary&nbsp; clone with&nbsp; a&nbsp; different&nbsp; phenotype.&nbsp; Rapid recognition&nbsp; of&nbsp; this&nbsp; condition&nbsp; is&nbsp; needed&nbsp; to tailor&nbsp; treatment&nbsp; decision&nbsp; and&nbsp; strategy&nbsp; for disease monitoring.&nbsp;&nbsp;</p>\r\n', 2),
(368, 365, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Breast cancer is the commonest cancer among females in most parts of the world, with an increasing incidence in developing countries(GLOBOCCAN 2000). In Malay-sia, it accounts for 30.4% of newly diag-nosed cancer and it is the commonest cancer in females of all ethnic groups (The National Cancer Registry 2002). The Ministry of Health (MOH) Malaysia, has advocated the practice of Breast Self Examination (BSE) as a screening tool for breast cancer since 1996. The Malaysian Clinical Practice Guidelines on manage-ment of breast cancer 2002,&nbsp;formulated by the Academy of Medicine and the Ministry of Health, is also in favour of monthly BSE for women aged 20 and above.</p>\r\n<p>\r\n	In the West however, the trend is moving away from BSE as there are controversies with regard to its practice. The Canadian Task Force on Preventive Health Care(CTFPHC) and the United States Preventive Services Task Force (USPSTF)states that there is insufficient evidence of benefit for or against BSE based on the review of a number of studies. One large trial, The Shanghai Trial(Harris and Kinsinger2002), claims that there is no evidence of reduction in mortality by performing BSE. The National Guideline Clearinghouse (NGC 2004) states that BSE causes undue anxiety, and increase in the number of physician visits for evaluation of benign breast biopsies in those who were taught BSE. The objective of this study is to determine how breast cancer was detected in a referral center, and how this information, along with the recent controversies, be used to justify the practice of BSE in our local setting.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Case notes of patients diagnosed with breast cancer from January 1996 to December 1997 were reviewed. All Malay-sian female patients were included. Those with missing files were excluded. Case notes were reviewed manually and entered into a pre-coded data collection form. Breast cancer presentation was divided into those with lump, skin manifestations (retracted nipple, peau d&rsquo;orange or ulcers) and those without any symptoms. The detection of the changes was categorised into those detected by the patients, medical personnel and mammogram. The average tumor diameter was recorded as docu-mented in the patient&rsquo;s file. The results were analysed using the Statistical Pack-age for Social Sciences (SPSS) software version 11.5.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 394 case notes were reviewed of which 366 were selected. Twenty-eight were excluded including 18 whose files were missing, five cases were foreigners, four had incomplete socio-demographic information, and one was a male patient.&nbsp; The age group most commonly affected were those between 40 to 49 years (39.6%) with a mean of 48.6 &plusmn;11.8 years. The vast majority (97.3%) of these patients detected the disease themselves. Medical staff detected 1.6% cases and 1.1% cases were detected by mammogram. The commonest mode of presentation is a breast lump (81.4%) followed by skin changes over the breast (17.5%). The mean tumour diameter on presentation was 4.7&plusmn;3 cm.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The peak age group for breast cancer presentation is similar to the study done by Yip and Ng 1996 and Hisham and Yip 2003. The common presenting symptom is a breast lump (81.4%), and most patients detected it themselves(97.3%). This is in contrast toan American study (Osteen et al 1994) where the number of women who presented with a breast lump was 42.2% and the rate of detection of the lump by the patients themselves was 43.9%. Although this figure appears lower, it must be emphasised that more than half of the lumps in that study were detected by screening mammogram; 56.1% compared to 1.1% in this study. This reflects the active role of screening mammogram in America. Another possible contributing factor is that women in this study probably detected the lump more easily because it was large. More Malaysian women pre-sented with tumours greater than 2cm (77.9%) compared to their counterparts in America (37.3%) and Singapore (63%) (Yip and Ng 1996).</p>\r\n<p>\r\n	Detection of the disease by mammo-gram in this study is 1.1%, which is much lower compared to the American study(Osteen et al. 1994). This is probably because the screening program in Malay-sia recommended by MOH does not in-clude mammogram as a mass-screening tool except in high-risk cases due to limited resources. In America, annual mammo-gram is done for all women 40 years and above as recommended by the American Joint Committee for Cancer (Leitch et al. 1997) based on meta-analysis of recent trials.</p>\r\n<p>\r\n	Although breast cancer was mostly detected by patients, it is uncertain whether it was detected on routine BSE or was merely an incidental finding due to the presence of a large lump but this infor-mation may be used to create awareness regarding the disease for early detection. There is no conclusive evidence that BSE reduces mortality but there are studies which have shown that the practice of BSE leads to detection of cancer in earlier stages and with less lymph node involve-ment (Huguly and Brown1981 and Hill et al. 1988).</p>\r\n<p>\r\n	Detection of disease based on mammo-graphic screening is very low in this study may be due to limited resources or due to the fact that it may not be used frequently enough as a screening tool to make an impact. Primary Care Physicians need to carefully select patients for mammogram to avoid under or over utilisation. Screening mammogram in Malaysia is recommended for women of 40 years and above since this is the peak age at presentation, instead of the current recommendation of 50 years of age. A large number of cases will be missed if screening is done 10 years beyond the peak age of presentation. Screening mammogram is costly but will probably be more economical than bearing the management cost of advanced stage disease with chemo and radiotherapy.</p>\r\n<p>\r\n	There is no doubt that mammogram is a sensitive tool (sensitivity &amp; specificity of 95 - 97%) and can detect lesions of up to 12mm, (Mushin et al. 1998) but BSE practice can be encouraged in countries and centres where there is cost and availability constrains for a mammogram. This can offer better options for treatment and a better quality of life if cancer is detected at an earlier stage.</p>\r\n<p>\r\n	This study, being retrospective, has limitations. Incomplete or missing case notes could not be analysed. The docu-mentation of the records was inconsistent, hence there was difficulty retrieving the relevant data. There may be a selection bias, as the sample studied represents the population attending a single hospital, hence may not give a true reflection of the disease presentation in the country.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	We would like to thank all staff of the Breast and Endocrine Clinic, Hospital Kuala Lumpur, Ministry Of Health for their assistance during this study.</p>\r\n', 2);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(369, 366, '<div>\r\n	<p align="left">\r\n		INTRODUCTION</p>\r\n	<p>\r\n		Chronic myeloid leukaemia (CML) is a malignant clonal myeloproliferative disorder affecting the pluripotent haematopoietic stem cell that can give rise to an increase of myeloid, erythroid cells and platelet in the peripheral blood, and myeloid hyperplasia of the bone marrow. The natural history of CML involves a triphasic phase from the chronic phase to an accelerated phase and then finally the blast phase.</p>\r\n	<p>\r\n		Chronic myeloid leukaemia is dis-tinguished from other chronic myelo-proliferative disorders by the presence of a distinctive chromosomal abnormality, termed the Philadelphia or Ph<sup>1</sup> chromo-some. This was the first consistent chromo-somal abnormality identified in cancer (Nowell 1960). The Philadelphia (Ph<sup>1</sup>) chromosome results from a reciprocal translocation between the long arms of chromosomes 9 and 22 [t(9;22)(q34;q11] and is present in at least 95 % of patients with CML (Nowell 1960). The Ph<sup>1</sup> chromo-some is also present in 5% of children and 20% of adults with acute lymphoblastic leukaemia (ALL) (Hagemeijer, 1987). In CML, there is a&nbsp; translocation of part of the long arm of chromosome 22 to the long arm of chromosome 9 and reciprocal translocation of part of the long arms of chromosome 9 to chromosome 22 (Ph<sup>1</sup> chromosome). In the BCR gene, the breakpoint occurs within a region of 5.8 kb, known as the major breakpoint cluster region (M-BCR) (Heisterkamp 1985). The majority of breakpoints occur between exons b2 and b3 or b3 and b4 of the M-BCR giving rise to b2a2 or b3a2 chimeric messenger RNA (mRNA) (Heisterkamp 1985).Both transcripts encode a p210 protein with prominent tyrosine kinase activity and transforming ability. This elevated tyrosine kinase activity plays an important role in the pathogenesis of the disease (Daley 1990).In the ABL gene, the breakpoint may occur anywhere within a region larger than 300kb upstream of the exon 1b, between exons 1b and 1a, or downstream from exon 1a.</p>\r\n	<p>\r\n		Due to this distinctive chromosomal and molecular abnormality, the detection of the t(9;22)(q34;q11) or the BCR/ABL re-arrangement is necessary for the diagnosis of CML and for monitoring of treatment response. The techniques of detection include conventional karyotyping and mole-cular analysis. In about 5% of cases diagnosed clinically as CML, no Ph<sup>1</sup> chromosome is observed (De Klein 1982). These cases have either more complex translocations involving other chromo-somes besides chromosome 9 and 22 or presence of cryptic BCR/ABL rearrange-ment (Kurzrock 1990). However, molecular analyses have shown that BCR/ABL is present in about half of these patients (Kurzrock 1990). Hence molecular analysis complement karyotyping by identifying abnormal clones undetected by karyo-typing.</p>\r\n	<p>\r\n		Conventional cytogenetics or karyo-typing, is the standard method to study proliferating cells in CML. Generally, they are based on analysis of up to 25 metaphases and can be used as a screening method to detect chromosome anomalies. The major advantage of chromosome analysis in CML is the possibility of detecting chromosome aberrations such as trisomy 8 and isochromosome 17, in addition to the Ph<sup>1</sup> chromosome (Nanjangud 1994).On the other hand, chromosome analyses are cumbersome, time consuming, labour intensive and dependent on high numbers of viable dividing cells.</p>\r\n	<p>\r\n		Fluorescence <em>in situ</em> hybridisation or FISH is rapidly becoming part of clinical practice in the workup of patients with haematological malignancies. FISH permits analysis of proliferating (metaphase cells) and non-proliferating (interphase nuclei) cells by detecting the location of specific nucleic acid sequences, using chromo-some specific DNA probes. These DNA probes contain a label (fluorescence) that allows their detection after hybridisation to the target of interest (Cox 1998). FISH has been used to reliably identify the BCR/ABL gene at the time of diagnosis (Cox 1998). Increasing usage of FISH probes have also successfully detected a variety of mole-cular-cytogenetics variants.Using FISH analysis, several studies have identified cases with additional phenomena to the translocation, such as large deletions of the derivative chromosome 9, which has been reported to have a worse prognosis (Sinclair 2000).</p>\r\n	<p>\r\n		The advantages of FISH are significant. It can be performed overnight and so it is particularly useful for situations which require diagnostic urgency. It is also easy to analyse large numbers of cells and the results are reproducible. It can be performed on various types of specimens, which include fixed cell pellets left over from cytogenetic studies on bone marrow or blood (Dewald 1998). They also work well with peripheral blood smears, touch preparations and paraffin embedded tissues (Paternoster, In press).</p>\r\n	<p>\r\n		Reverse transcriptase polymerase chain reaction (RT-PCR) is another method of molecular analysis in detecting the BCR/ABL translocation. It is a highly sensitive technique for the detection of BCR/ABL mRNA. Using PCR, one can detect leukaemia specific mRNA se-quences extracted from as few as one cell. The technique consists of two parts. The first part involves the synthesis of cDNA (complementary DNA) from RNA by reverse transcription. The second part involves the amplification of a specific cDNA by PCR. In our study, we have considered PCR as the gold standard.</p>\r\n	<p>\r\n		Several studies have addressed the issue of which is the clinically most useful approach for the detection of BCR/ABL rearrangement in Ph<sup>1</sup> positive patients. Cox et al in 1998 reported that the sensitivity of PCR and FISH related to Ph<sup>1</sup> positive cases were 97% and 100% respectively with 5% false positive results by PCR. In another study on 40 samples of Ph<sup>1</sup> positive related disorders, Tbakhi et al (1998) found that FISH and PCR were positive in 100% of CML samples. In these two studies, both of them had utilised conventional cytogenetics (karyotyping) as their gold standard.&nbsp; Both of these studies had concluded that either FISH or PCR procedure offers a specific, cost effective and faster alternative to karyotyping. Both of these studies had analysed all the Ph<sup>1</sup> related disorders which include CML, acute lymphoblastic leukaemia and acute myeloid leukaemia.</p>\r\n	<p>\r\n		The present study aims to evaluate the role of FISH in detecting the BCR/ABL rearrangement in clinically diagnosed CML patients. A comparative analysis between FISH (molecular cytogenetics) and conven-tional karyotyping were carried out utilising PCR as our gold standard. The signal patterns obtained using the BCR/ABL ES (Extra Signal) FISH probes were also studied.</p>\r\n	<p>\r\n		<strong>METHODOLOGY AND MATERIALS</strong></p>\r\n	<p>\r\n		This is a cross sectional study on all cases&nbsp; diagnosed with CML at various stages of therapy and those suspected of CML from Hospital Universiti Kebangsaan Malaysia (HUKM) recruited between January 2003 and December 2004. Cytogenetics, PCR and double colour ES FISH analyses were performed on the samples for the presence of BCR/ABL gene rearrangement.&nbsp; Routine diagnostic procedures for these patients consisted of morphology and differential white blood cell counts on bone marrow and peripheral blood. Diagnosis of CML was made by clinical findings, bone marrow aspirate and neutrophil alkaline phospahatase score. Patients with other forms of myeloproliferative disease including Acute Lymphocytic Leukemia (ALL) and Acute Myeloid Leukemia (AML) were not included.</p>\r\n	<span>&nbsp;</span>\r\n	<p>\r\n		<em><span>&nbsp;</span>Conventional Cytogenetics (Karyotyping)</em><span>&nbsp;</span></p>\r\n	<span>&nbsp;</span>\r\n	<p>\r\n		Samples of bone marrow were cultured and harvested following standard protocol (Barch 1997). Details of the karyotype were reported according to ISCN 1995.</p>\r\n	<p>\r\n		<em>Fluorescence in situ Hybridisation (FISH)</em></p>\r\n	<p>\r\n		ES (extrasignal) FISH analysis was performed on the same bone marrow samples harvested for cytogenetics examination and following manufacturer&rsquo;s protocol (Vysis Inc. Downers Grove, Illinois, USA). Hybridisation signals were evaluated using BX60 fluorescence microscope equipped with appropriate filters for simultaneous visualization of fluorescein isothiocynate (FITC) and tetra-methylrhodamine isothiocynate (TRITC) fluorescence.</p>\r\n	<p>\r\n		<em>Reverse Transcriptase - Polymerase Chain Reaction (RT-PCR)</em></p>\r\n	<p>\r\n		In this study, we considered PCR as the gold standard in the diagnosis of CML. We adopted strict precautions and conditions to avoid contamination.<br />\r\n		<br />\r\n		<em>Total RNA isolation and cDNA synthesis</em></p>\r\n	<p>\r\n		Mononuclear cells from fresh bone marrow aspirates were isolated by Histopaque density gradient centrifugation. The mononuclear layer was aspirated into a 5 mL tube and stored at &ndash;80<sup>o</sup>C. The&nbsp; RNA extraction was performed using the acid guanidinium thiocyanate - phenol - chloro-form extraction (AGPC) method.</p>\r\n	<p>\r\n		Complementary DNA (cDNA) synthesis was done using 5X RT buffer, MgCl<sub>2</sub>, PCR nucleotide mix, Rnasin, random hexamers and Moloney murine leukemia virus reverse transcriptase (M-MLVRT, Promega Corp). RNA sample was initially transferred into a PCR tube and heated at 70<sup>o</sup>C for 10 minutes using a thermocycler. After a quick chill in ice, cDNA mix was then added to the RNA sample. PCR was performed on the mixed cDNA sample consisting of 1 cycle at 37<sup>o</sup>C for 90 minutes and 65<sup>o</sup>C for 10 minutes. The sample was then stored at &ndash;20<sup>o</sup>C.</p>\r\n	<p>\r\n		<em>Reverse Transcriptase - Polymerase Chain Reaction (RT-PCR)</em></p>\r\n	<p>\r\n		Master mix for PCR (distilled water, 10x PCR buffer, primers, 10mM of PCR nucleotide mix and 25 mM of MgCl<sub>2</sub>, Promega Corp) was added to the PCR tube along with cDNA sample. After a hotstart of 1 min at 94<sup>o</sup>C, 0.5 uL of Taq DNA polymerase was added to each sample. PCR was performed on an automated thermocycler which consisted of 35 cycles at 94<sup>o</sup>C for 1 minute, 65<sup>o</sup>C for 50 seconds, 72<sup>o</sup>C for 1 minute, and final extension at 72<sup>o</sup>C for 10 minutes. Reaction products were electrophoresed on a 1.5% agarose gel with ethidium bromide staining for 45 minutes at 80V. The products were then visualised under ultraviolet light.</p>\r\n	<p>\r\n		<strong>RESULTS AND DISCUSSION</strong></p>\r\n	<p>\r\n		Eighteen cases were studied by conventional karyotyping, FISH and RT-PCR. The results are presented in Table 1. BCR/ABL mRNA was detected in 13 of</p>\r\n	<p>\r\n		these samples (72%). The remaining five samples did not show presence of BCR/ABL mRNA. Eleven patients (61%) were Ph<sup>1</sup> positive (Table 1 and Figure 1) and all of them showed presence of BCR/ABL mRNA by PCR. Two out of the remaining seven samples did not have any metaphase and showed very few cells respectively. However, these two samples were proven mRNA positive by PCR. The other five samples showed normal karyotype with no abnormalities seen. For conventional karyotyping, the sensitivity, specificity, positive predictive value (PPV) and negative predictive value (NPV) were 85%, 100%, 100% and 71% respectively (Table 2). The accuracy for conventional karyotyping method was 88%. The lower sensitivity was obviously due to a smaller number of samples utilised in our study. A previous larger study has quoted sensitivity of at least 95% (De Klein 1982). In patient 9, there was presence of an extra copy of chromosome 8 (trisomy 8) and iso-chromosome 17q (Figure 3). Seventeen samples were evaluable for FISH analysis. FISH was not performed on one sample due to insufficient material. Twelve samples (71%) showed presence of BCR/ABL fusion gene by FISH analysis(Table 1 and Figure 2) and all of these twelve samples showed presence of BCR/ABL mRNA by PCR. The remaining five samples did not show presence of the fusion gene by FISH. The sensitivity, specificity, PPV and NPV for FISH were all 100% (Table 3). The accuracy for FISH analysis was 100%. Previous studies have quoted sensitivities of FISH between 80% and 100% (Cox 1998, Tkachuk 1990). FISH had also confirmed presence of two BCR/ABL fusion genes in one patient. The cytogenetic analysis of this patients&rsquo; sample also showed presence of two Ph<sup>1</sup> chromosomes (Figure 3 and Figure 4)<strong>. </strong></p>\r\n	<p>\r\n		From our study, we detected the BCR/ABL fusion gene in 12 of 17 samples analysed by FISH method (Table 4). The possibility of a false positive result by FISH was ruled out because the PCR finding on this sample was BCR/ABL mRNA positive.</p>\r\n	<p>\r\n		Based on results of conventional cytogenetics (karyotyping) in our study, we acknowledge that conventional cyto-genetics (karyotyping) has its limitations. We could not obtain any metaphase spread in one of our samples and in another sample, only very few cells were present. The proportions of metaphases identified in cytogenetics depend on several factors, such as the number of metaphases analysed and the method used for obtaining metaphases. These metaphases only represent a small proportion of the total cells of a sample. However, conventional cytogenetics (karyotyping) has its strength because it is able to detect additional nonrandom chromosomal changes.</p>\r\n	<p>\r\n		In conclusion, the ES fluorescence <em>in situ</em> hybridisation (FISH) technique is a highly sensitive and accurate method for detection of the BCR/ABL fusion gene. Despite several limitations, conventional karyotyping, however, will remain an important investigation in the work up of suspected CML patients since there is a possibility in detecting chromosomal aberrations in addition to the Ph<sup>1</sup> translocation. Therefore, conventional cyto-genetics (karyotyping) and FISH are complementary techniques and their results should be interpreted together with clinical information and other molecular studies.</p>\r\n</div>\r\n', 1),
(370, 367, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Patient satisfaction is the patient&rsquo;s perception of care received compared with the care expected. Patients base their expectations on their own encounters with behaviours of nurses (Aiello et al 2003). According to Eriksen (1995), critical attributes of patient satisfaction with quality nursing care are reliability, tangibles, responsiveness, empathy and information giving. Reliability is defined as being &ldquo;dependable, accurate, timely, competently, done right the first time.&rdquo; &ldquo;Assurance includes &ldquo;trust, problem is in good hands, credibility, security, and reassurance&rdquo;.&nbsp; Tangibles are &ldquo;physical appearance of facilities staff.&rdquo; Responsiveness consists of &ldquo;promptness, accessibility.&rdquo; Empathy relates to &lsquo;individualised attention, kind, courteous, understanding, friendly, able to listen, unhurried attention.&rdquo; Information giving is &ldquo;keeping patient informed&rdquo; and &ldquo;explaining to patient&rdquo;. During hospitalisation, patient satisfaction represents a balance between the patient&rsquo;s perception and expectation of their nursing care (Han et al 2003). The patient&rsquo;s perception of&nbsp; quality nursing care also include caring, compassion, competence, confidence, conscience and commitment in the delivery of care (Gunther et al 2002). It is interpreted as a therapeutic response, which leads to speedy recovery among post-cardiac surgery patients (Doering et al 2002; William 1998).</p>\r\n<p>\r\n	The achievement of patient satisfaction among post cardiac surgery patients is specific to the interventions implemented by the nurse. The four domains of outcomes have been identified as technical competence, information giving, assurance and empathy (Larrabee et al 2001). The post cardiac surgery patients require constant vigilant nursing care, as their condition can be life threatening (Doering et al 2002). The types of cardiac surgeries include coronary artery bypass graft (CABG), valve surgery, correction of atrial septal defect (ASD), ventricle septal defect (VSD), tetralogy of Fallot (TOF), pulmonary atresia and aortic aneurysm. Post cardiac surgery patient satisfaction is pivotal to the achievement of optimum clinical outcomes, speedy recovery, reduced length of stay and avoidance of recurrent admission (Larrabee et al 2001). On the other hand, patient dissatisfaction would result in poor compliance with the treatment regimen, premature self-termination of care plan and seeking care outside the plan. Such patients would portray anger and verbal abuse (Jafar et al 2003). Achieving optimum patient satisfaction with quality nursing care has been the primary focus of nurses.&nbsp;</p>\r\n<p>\r\n	It is with this intent that the researcher conducted a study on post cardiac surgery patient satisfaction with quality nursing care in the surgical wards of IJN. The objective of this study was to identify factors in quality nursing care that determined patient satisfaction.</p>\r\n<p>\r\n	<strong>SUBJECTS AND METHODS</strong></p>\r\n<p>\r\n	A descriptive study was undertaken at IJN. This study was conducted from January 2005 till March 2005. A conceptual framework was formulated for the purpose of having an overview of the critical attributes (Figure 1).&nbsp; Primary data were collected using NURSQUAL, a fully structured questionnaire, to measure the four domains in relation to technical competence, information giving, assurance and empathy (Larrabee et al 2001). There were two sets of questions; section A consisted of 33 items to measure quality nursing care and section B comprised six questions with regard to demographic data. In section A, the above questions were scored with a seven-point Likert-type scale. Scores were rated as follows: one-strongly disagree and seven-strongly agree, whereas section B used a nominal scale. Fifty two respondents who fulfilled the inclusion criteria were recruited from the surgical wards (Mawar and Cempaka) of&nbsp; IJN.</p>\r\n<p>\r\n	The inclusion criteria of the respondents were post cardiac patients more than 18 years old, elective cardiac surgery CABG and valve surgery and emergency cardiac surgery (CABG) and valve surgery without complication. The exclusion criteria of the respondents were those undergoing any emergency cardiac surgery (CABG) and valve surgery with complications, critically ill and nursed in high dependency unit.</p>\r\n<p>\r\n	The research instrument was a self-administered questionnaire. The questions relating to the four domains are listed in Figure 2, Figure 2A, and Figure 2B</p>\r\n<p>\r\n	Ethical approval was undertaken and permission obtained from the ethics committee of the Faculty of Medicine, University Kebangsaan Malaysia (UKM) and Institute Jantung Negara (IJN). The study was carried out in accordance with the ethical principles and guidelines of UKM and IJN. Confidentiality of the respondents were assured. Research questionnaires were given and collected by the researcher on the same day, to prevent respondents from discussing their answers among themselves or with others such as family members, nurses or visitors.</p>\r\n<p>\r\n	Statistical analysis was done using the statistical package for social science (SPSS) windows, version 11.5.&nbsp; Demographic data of respondents were analysed by descriptive statistics of frequency. Statistical methods included Pearson correlation coefficient, the differences of mean between patient satisfaction with technical competence, information giving, assurance and empathy. A multiple linear regression test was utilised to identify the attributes on patient satisfaction with quality nursing care (Burns &amp; Bush 2003).</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Respondents&rsquo; demographics data consisted of gender, age, ethnicity, marital status, highest education level and occupation, as shown in Table 1.</p>\r\n<p>\r\n	Eighty three percent of patients agreed that the nurses were technically competent, 83% that they gave adequate information, 87% that they provided assurance, and 94% that they showed empathy (Table 2).</p>\r\n<p>\r\n	There was a strong correlation between technical competence, information giving, assurance and empathy with questions on &ldquo;how would you rate your satisfaction with overall nursing care received during stay in hospital?&rdquo;, &ldquo;would you comply to the nurses advise regarding your care at home?&rdquo;, &ldquo;would you come back to this hospital to seek treatment if needed?&rdquo; and &ldquo;do you have intention of recommending this hospital to your family member, relatives and friends?&rdquo;. (Table 3)</p>\r\n<p>\r\n	The strongest predictors of patient satisfaction were assurance (beta coefficient of 0.557) and technical competence (beta coefficient of 0.280) (Table 4)</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	The findings from this study were consistent with those previously published. Patient satisfaction with regards to the four domains of quality nursing care may vary but they are interrelated to one another (Aiello et al 2003; Jafar et al 2003; Uzun et al 2001). The results of this study highlighted the importance of technical competence, information giving, assurance and empathy in the holistic management of the post cardiac surgery patient during hospitalisation.</p>\r\n<p>\r\n	The study found a significant association between gender and the elements of assurance and empathy. Men were found to be more satisfied than women. The reason for this gender differences in our study was not specifically explored in-depth.&nbsp; According to Lumby et al (2000), women possessed negative perceptions of assurance than men. Valentin et al (2005) concluded similar findings in relation to gender which may be true for our population.</p>\r\n<p>\r\n	According to Uzun (2001), patients&rsquo; aged &ge; 50 years gave high scores for nursing care compared to patients aged &lt;50 years.&nbsp; The results of the present study support this finding and that of another study where older patients were found to be more compromising and contented (Staniszewska et al 1999), particularly with regards to the domains of technical competence, assurance and empathy.</p>\r\n<p>\r\n	The results of this study also showed a significant positive correlation between the elements of technical competence, information giving, assurance and empathy, and the overall satisfaction with nursing care, willingness to comply with nurses advice regarding their care at home, willingness to come back to the hospital to seek treatment if needed and intention of recommending the hospital to family members, relatives and friends (p value &lt; 0.01 and 0.05). Jacox et al (1997) found a strong relation between technical skill and quality nursing care provided, which is in keeping with our findings.</p>\r\n<p>\r\n	For element of information giving, a positive association exist between patient compliance to the nurses advice regarding their care at home. Jafar et al (2003) also reported similar findings with respect to patient&rsquo;s information, satisfaction and hospital revisits. Similarly, for element of empathy, a positive association was also noted. Lumby et al (2000) reported that empathy had a significant relationship with patient satisfaction. It is also unclear how the weighting of each dimension could be determined, since there will be differences in their relative importance for each patient as each person has a unique frame of reference and a set of personal norms for making judgments about nursing care (Valentin et al 2005). There will be differences based on patient&rsquo;s attitudes to receiving care, their knowledge of the services available, their previous experience and reports that they obtained from others.</p>\r\n<p>\r\n	Indeed the most valuable element in our study was assurance provided by the nurses. &ldquo;Assurance&rdquo; made the strongest statistically significant unique contribution to the prediction of patient satisfaction. From an organization structure perspective, nurses who use rules, procedures and instructions positively would influence patient satisfaction (Lumby et al 2000). Since IJN has been established as a cardiac referencing hub,it has been used as a benchmark for other hospitals in Malaysia to emulate their own cardiac centre. The organization has experts in every sector of their management, including nursing. Post cardiac surgery patients are more satisfied with nurses who incorporate organisational and professional guidelines as their tool for implementing quality nursing care. Therefore, organisation must consistently include the latest guidelines and standards in their rules, procedures and instructions to meet the demands of globalisation and evolution in nursing care (Han et al 2003).&nbsp;</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, this study has demonstrated the importance of nurses being sensitive and knowledgeable of four major domains, which are technical competence, information giving, assurance and empathy among the post cardiac surgery patients.&nbsp; Of these, assurance from the nurses has been found to be of top priority and a crucial factor in determining patient satisfaction with quality nursing care.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	The authors thank Associate Professor Dr. Aliah Hanim M. Salleh (UKM, Bangi Campus) for her review of the research proposal.</p>\r\n<p>\r\n	The authors also extend their gratitude to the Management of IJN, post cardiac surgery patients and nurses for their cooperation in responding to the survey; without their help, this work could never have been done.</p>\r\n<p>\r\n	Heartfelt thanks to Prof. Dr. Lokman Saim, Dean Faculty of Medical UKM for giving permission for this study to be published.</p>\r\n<p>\r\n	We wish to extend our sincerest thanks to Puan Raiah Hj. A. Rahim Head of Nursing Department for her continuous support.</p>\r\n', 1),
(371, 368, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n</div>\r\n<p>\r\n	Lactate is a gluconeogenic precursor that is rapidly metabolized, particularly during starvation or when there is increase in body catabolism. A study done by Thomas and Alberti showed that in Non Insulin Dependant Diabetes Mellitus (NIDDM) patients who were given Ringer&rsquo;s lactate&nbsp; in amount of 29-44 mmol during surgery had an increased in mean plasma glucose of 7.5 mmol/l when compared to those not given intravenous fluids amounting only 2.1 mmol/l (Thomas and Alberti 1978).Therefore, higher insulin doses may be required for diabetic patients receiving Ringer&rsquo;s lactate during the peri-operative period. In another study, it was shown that the infusion of Ringer&rsquo;s lactate at a rate of 25 &micro;mol/kg/min over three hours in non diabetic patients did not alter plasma glucose production (Jenssen et al. 1989). However, there is no study yet to compare the effect of giving normal saline to patients with NIDDM when compared to Ringer&rsquo;s lactate. With this interest in mind, a prospective randomized study was done to evaluate the effect of Ringer&rsquo;s lactate in comparison with normal saline on the glycaemic control in patients with NIDDM.&nbsp; The aim ofthis study was to compare the intraoperative glycaemic control in patients with NIDDM when given either normal saline or Ringer&rsquo;s lactate.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This was a prospective&nbsp; randomised controlled single blind study carried out in Hospital Sultanah Aminah, Johor Bahru. After obtaining institutional approval and informed consent, 50 ASA II patients with NIDDM aged 21-70 years undergoing elective or emergency surgical procedures under spinal or lumbar epidural anaesthesia lasting more than one hour were included in the study. Exclusion criteria included patients who have impaired renal or liver function, allergy to the regime solution used and those patients where spinal or lumbar epidural were contraindicated.</p>\r\n<p>\r\n	All patients were assessed pre-operatively the day before and no premedication was served. An infusion of&nbsp; dextrose 5% at 100 mls/hr was administered once patients fasted until their arrival to the operation room. All oral hypoglycaemic agents were omitted on the day of operation. In the operation theatre, patients were randomized into two groups using a table of random sampling. Patients in Group 1 received normal saline and patients in Group 2 received Ringer&rsquo;s lactate. Both regimes were infused at 100 mls/hr using an infusion pump. Spinal or lumbar epidural anaesthesia was then performed under aseptic technique using a 27G Pencan<sup>&reg;</sup> or 18G Tuohy needle. Throughout the operation, all patients were monitored with a pulse oximeter, electrocardiogram and a non invasive blood pressure monitoring device. All patients received supplemental oxygen at 6 l/min via a simple face mask.</p>\r\n<p>\r\n	Blood glucose levels were measured using a glucometer by the pinprick method. Levels were measured at baseline (before performing spinal or epidural blockade), at 45 minutes intra operatively, at 30 minutes, and four hours postoperatively. Other parameters recorded include duration of anaesthesia, volume of regime fluids infused and rescue medication given if any.</p>\r\n<p>\r\n	Duration&nbsp; of&nbsp; anaesthesia is defined&nbsp; as the time interval from the&nbsp; onset of neural blockade till&nbsp; completion of&nbsp; surgery.&nbsp; Rescue&nbsp; action&nbsp; will&nbsp; be&nbsp; taken&nbsp; if&nbsp;&nbsp; the&nbsp; blood glucose&nbsp; level&nbsp; is below&nbsp; 2.2 mmol/l or more than 15.0 mmol/l, according to the British&nbsp; National&nbsp; Formulary 1997;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<table border="0" cellpadding="0" cellspacing="0" style="width:100.0%;" width="100%">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:35.08%;">\r\n				<p>\r\n					&lt; 2.2 mmol/l</p>\r\n			</td>\r\n			<td style="width:64.92%;">\r\n				<p>\r\n					50 mls dextrose 50% to be given.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:35.08%;">\r\n				<p>\r\n					2.2-15.0 mmol/l</p>\r\n			</td>\r\n			<td style="width:64.92%;">\r\n				<p>\r\n					No action.</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:35.08%;height:14px;">\r\n				<p>\r\n					&gt; 15.0 mmol/l</p>\r\n			</td>\r\n			<td style="width:64.92%;height:14px;">\r\n				<p>\r\n					4&uuml; insulin/hr and adjusted accordingly.</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Hypotension is defined as a drop in 20% of systolic blood pressure from baseline and is treated with boluses of 5 ml/ kg of the infused fluids for the groups respectively. Boluses of ephedrine 6mg every 10 minutes will be given if hypotension persisted despite adequate boluses of fluids given. All patients were kept in the recovery room for the duration of the study.</p>\r\n<p>\r\n	Data was analysed using paired Student&rsquo;s t test and Chi- square test where appropriate. A &lsquo;&rho;&rsquo; value of &lt; 0.05 was used as the level of statistical significance.</p>\r\n<p align="left">\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Demographic data and patient&rsquo;s variables are shown in Table 1. Both groups were comparable in terms of age, weight, sex, duration of anaesthesia and volume of intravenous fluids given.</p>\r\n<p>\r\n	The mean blood glucose levels for the two groups is shown in Table 2. There was an increase in blood glucose levels with time in both groups, except at 45 minutes intraoperatively in Group 1. There were significant differences in the mean blood glucose level between Group 1 and 2 except at baseline. The increase in the mean blood glucose level at 4 hours postoperatively when compared to baseline is greater in Group 2 which was 1.50 mmol/L (19%) versus 0.96 mmol/L (13%) in Group 1. However, this difference was not significant (&rho;&gt;0.05). There was no difference in the increase of mean blood glucose levels in both groups in the intraoperative period when compared to baseline. Whereas, there was significant increase seen in the postoperative period.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Diabetes mellitus is the commonest endocrine disorder encountered in anaesthesia and its incidence is increasing (Milaskiewicsz and Hall 1992, Scherpereel and Tavernier 2001). Ninety percent of diabetic patients are NIDDM and this is attributed to the sedentary lifestyle, genetic predisposition and diet. It is expected that the number of people in the world with diabetes will double in the next decade (Hall et al. 1999). Due to advances in medical and surgical therapy, it is likely that diabetic individuals have a greater chance of undergoing surgery today such as vitrectomy, cataract extraction and ulcer debridement with good outcome (Hirscjh et al. 1991).</p>\r\n<p>\r\n	In this study, we are looking specifically on the type of solutions in perioperative glycaemic control. Hartmann&rsquo;s solution is used interchangeably as Ringer&rsquo;s lactate in daily use. It is often regarded as the anaesthetic panacea of fluid resuscitation. This crystalloid formulation is routinely employed as the rehydration and maintenance solution of choice in the operation theatre (White and Goldhill 1997). Often, it is given to diabetic patients as an alternative to glucose solutions. Lactate is a gluconeogenic precursor, and the rate of gluconeogenesis may be enhanced in diabetes, particularly in situation of stress such as surgery. Infusion of lactate may have unwanted metabolic effects in diabetic patients.</p>\r\n<p>\r\n	There are limited studies on glycaemic control in diabetic patients using Ringers lactate. In a study by Thomas and Alberti, they made a comparison between three groups of patients. Group 1 (normal patients receiving one litre Ringer&rsquo;s lactate) has a significant increase in blood glucose level (55%). Group 2 (NIDDM&nbsp; patients&nbsp; not&nbsp; receiving&nbsp; any intravenous fluids)&nbsp; had only 22% significant increase in blood glucose level. Group 3 (NIDDM patients receiving one litre Ringer&rsquo;s lactate) produce the largest significant increase in blood glucose level at 88% (Thomas 1978). When the percentage increase in plasma glucose was compared in diabetic patients with or without Ringer&rsquo;s lactate solution, the difference was highly significant. Demographically, the above study has a similar pattern to this study, except that the Thomas and Alberti study has a smaller sample (five samples in each group), being premedicated and his patients were absolutely fasted at least for six hours with no dextrose 5% infusion administered. In this study, all patients were not premedicated and were given normal maintenance at 100 mls/hour of dextrose 5% to prevent the occurrence of hypoglycaemia. Patients in Group 2 had an increase in mean blood glucose level&nbsp; of 1.50 mmol/L (19%) in comparison with patients in Group 1 which had only 0.96 mmol/L (13%) which was not statistically significant. However, when compared to the Thomas and Alberti study, this study shows a marked less increase in mean blood glucose level (19% compared to 88%).</p>\r\n<p>\r\n	There are other confounding factors which may influence peri-operative glycaemic control such as catabolic factor, type of solutions and type of surgery. Fasting with a maintenance of intravenous dextrose 5% solution gives a better glycaemic control preoperatively than those without it. This has been shown in another study by Rauoules, Lugrin and Boussofara (1994), in which an infusion of dextrose 5% at 125 mls/hr maintains the glycaemic control perioperatively. Furthermore, normal saline is a non lactated solution, therefore, a smaller increase in mean blood glucose level is expected when compared to Ringer&rsquo;s lactate.</p>\r\n<p>\r\n	In emergency cases, the rate of increase in glucose disappearance in response to an increase in insulin concentration is impaired in people with NIDDM (Turk 1995). Usually, in emergency cases, patients are not well optimised and it is assumed the stress response is much greater. Thus, most of the time the glycaemic control is poor to start with. This will invariably affect the glycaemic control perioperatively. Even though it has not been specifically analysed in this study, it may contribute to a significant higher increase in mean blood glucose level seen in Group 2.</p>\r\n<p>\r\n	In conclusion, we found that the glycaemic control was worsened with the use of Ringer&rsquo;s lactate solution. Therefore, we recommend that it is better to avoid lactated solution in patients with NIDDM, especially when the preoperative glycaemic control is already poor to start with.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	We would like to thank the Deputy Dean and staff of Post Graduate Studies and Research, Faculty of Medicine UKM, for allowing us to submit this manuscript for publication.</p>\r\n', 2),
(372, 369, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Non-specific viral febrile illness is a common encounter in an outpatient clinic (Teng et al. 2003). It is an important entity especially in countries where dengue infection is prevalent. This is because one of the common presenting symptoms of dengue infection is non-specific febrile illness (Gibbons and Vaughn 2002). Many clinicians would report and manage a case of acute non-specific viral febrile illness with thrombocytopaenia as dengue infection based on clinical assessment alone (Audit of Dengue notification in Pusat Perubatan Primer HUKM 2004).</p>\r\n<p>\r\n	However, dengue infection is only one of the many causes of acute febrile thrombocytopaenia. Other causes of febrile thrombocytopaenia include scrub typhus, chikungunya fever, infectious mono-nucleosis, malaria, typhoid fever, leptospirosis&nbsp; and acute human immuno-deficiency virus conversion disease (Vaughn and Green. 2000). Reporting every case of acute non-specific viral febrile thrombocytopaenia as dengue infection would lead to over reporting. Furthermore, managing all cases of acute non-specific viral febrile illness with thrombocytopaenia as dengue infection may not be appropriate as acute non-dengue viral febrile illness with thrombocytopaenia may differ in their natural history and treatment from dengue infection. Some require less intensive monitoring as in chikungunya fever; some may need antibiotics such as scrub typhus and typhoid fever; some may have other serious outcome such as splenic rupture in infectious mononucleosis and acute human immunodeficiency virus conversion disease. Hence it is important to differentiate patients presenting to the outpatient setting with acute non-dengue febrile thrombocytopaenia from those having dengue infection. Deparis et al in 1998 reported 15.5% of acute non-dengue febrile patients have thrombocytopaenia. There is no published data on the prevalence of such an entity among patients presenting with acute non-specific viral febrile illness. Knowing its prevalence will help us estimate the magnitude of the problem. The objective of this study was to determine the prevalence of non-dengue febrile thrombocytopaenia among febrile patients presenting to the outpatient clinic.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHOD</strong></p>\r\n<p>\r\n	This was a cross sectional study of a group of patients attending the Primary Care Center of Hospital Universiti Kebangsaan Malaysia (HUKM) and Batu 9 Health Clinic Hulu Langat, from May to November 2003. Consecutive patients who fulfilled the study criteria were invited to participate in the study. The inclusion criteria were age 12 years and above with a history of fever of less than two weeks. Fever was defined as recorded oral temperature of more than 37.5<sup>o</sup>C. Patients were excluded if there was an apparent localised source of infection e.g. urinary tract infection or acute abdomen. Any illness with pathognomonic clinical features e.g. varicella infection, measles, rubella, scarlet fever and obvious features of dengue haemorrhagic manifestations were also excluded. Consents were taken from all eligible patients before enrolling them into the study.</p>\r\n<p>\r\n	Full blood count examination was carried out for every patient seen. Patients were assumed to have dengue infection if thrombocytopaenia (platelet count of &le; 150X10<sup>9</sup>/L) was detected. They were managed as per protocol for management of dengue infection. All the other patients with normal platelet count were called back on day 5 of illness for a repeat full blood count so as not to miss any possibility of thrombocytopaenia. Dengue serology was done for all patients on day five of illness, as early blood sampling of less then day five of illness might yield false negative results. If the patients require admission anytime during the study, the data was than collected from hospital records.</p>\r\n<p>\r\n	Dengue serology tests were run with PanBio Rapid Immunochromatography method in the virology laboratory, HUKM. The methods, reagents used and interpretation of results were as described by the manufacturer. Primary acute dengue infection is defined as a positive serology test of IgM alone. Secondary infection is defined as a positive IgG with or without a positive IgM. Collectively, both primary and secondary infections constitute acute dengue infection. A negative result is when both IgG and IgM are negative.</p>\r\n<p>\r\n	<em>Statistical analysis and sample size calculation</em></p>\r\n<p>\r\n	Analysis of the data was carried out using SPSS version 11. The denominator is the total number of cases presented as non-specific febrile illness. Calculated minimal sample size of 61 patients was required, based on the estimated prevalence of non-dengue thrombocytopaenia of 20% (95% confidence interval; worst estimated of 10%) among patients presenting with acute non-specific viral febrile illness. Ethics approval was obtained from the research and ethics committee of the Medical Research Center UKM.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	153 patients were screened, 49 did not give consent for the study and 17 were excluded for not fulfilling the criteria. Fourteen patients defaulted follow up as they claimed to be well and refused reassessment. Complete data was available for 73 patients. Mean age of the patients in this study was 27.3 years (27.3, SD&plusmn;11.1). Their baseline characteristics are presented in table 1. Figure 1 summarises the number of patients enrolled in this study. The majority of the patients enrolled were Malay (72.6%).</p>\r\n<p>\r\n	Thirty five of 73 patients were found to have thrombocytopaenia. Hence, the prevalence of thrombocytopaenia among acute non-specific febrile illness was 47.9%. Among these patients, 40% (14) were serologically negative for dengue infection, and only 60% (21) were diagnosed to have dengue infection. The overall prevalence of acute non-dengue viral thrombocytopaenia among patients presenting with non-specific viral fever is therefore 19.2%.</p>\r\n<p>\r\n	Of the remaining 38 patients with the normal platelet count, three were tested positive for dengue serology. Therefore, total number of dengue infection was 24 and hence the prevalence of dengue infection among patients presenting with non-specific viral fever was 32.9%.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	This study confirmed that thrombo-cytopaenia is common among adult patients with non-specific febrile illness. About half of them (47.9%) had thrombocytopaenia. A significant proportion of patients with thrombocytopaenia (40%) were serologically negative for dengue infection. Hence, equating all patients with thrombocytopaenia as having dengue infection may lead to over diagnosis. However, one has to be cautious when interpreting this data. Panbio Rapid Immunochromatography test kit was used as a gold standard for diagnosis in this study. Serologically negative result for dengue does not rule out dengue infection. Antigen detection by polymerase chain reaction, viral culture or paired serology should be used as the gold standard for diagnosis (WHO 1997).Therefore, even though Panbio Rapid Immunochromato-graphy test kit has very good sensitivity and specificity, false positive and negative test results do occur. In reference to a local study by Lam et al., the false negative rate of this Panbio Rapid test was 0% (sensitivity of 100%) and the false positive rate was 11% (specificity of 89%) (Lam and Devine 1998). Whereas, Vaughn et al. demonstrated that 80% of dengue patients would show a diagnostic level of secondary dengue infection by day 1 of defervescence using haemagglunition-inhibition test (HAI, in detecting IgG) which gave a false negative of 20%. Day of defervescence was defined as the day when the temperature dropped below 38<sup>o</sup>C which commonly occurs around day 5 to 7 of illness (Vaughn et al. 1997). In the present study, the false negative rate for serological diagnosis using Panbio Rapid Immunochromatography test was probably less then 20%. Considering the rate of false negative as 20%, there would still be 11 patients (33.3%) with seronegative results and the prevalence of acute non-dengue viral thrombocytopaenia among patients presenting with non-specific viral fever would therefore be 15.1%.</p>\r\n<p>\r\n	Deparis et al reported that 15.5% of acute non-dengue febrile patients have thrombocytopaenia (Deparis et al. 1998). However, patients from this study were among those suspected to have dengue infection, and not all patients with acute non-specific febrile illness were from an outpatient setting. From this data, it is evident that non-dengue thrombo-cytopaenia is a significant entity that should be differentiated with dengue infection. It is important to differentiate between these two groups, as acute non-dengue febrile thrombocytopaenia might be different in their natural history from those with dengue infection. At present, all patients with acute non-specific febrile illness and thrombo-cytopaenia are assumed to have dengue fever, and they are managed as such. This may not be the appropriate management as the other causes of febrile thrombo-cytopaenia require different management strategies.</p>\r\n<p>\r\n	In this study, data were collected prospectively in order to standardise data collection and to minimise incomplete data. The patients selected were all from outpatient clinic with non-specific viral febrile illness and the result confirmed that dengue infection indeed is a common problem among them during the non-epidemic season, whereby the prevalence was 32.9%. The prevalence of dengue infection is consistent with that of other studies, where dengue infection is endemic, ranging from 21%-35% (Trofa et al. 1997, Deparis et al. 1998, Akram et al. 1998, Kalayanarooj et al. 1998). However, 32.8% of the patients eligible for this study were unwilling to participate. They were probably suffering from a milder form of non-dengue viral fever. Hence, the true prevalence might be lower then 32.9%. During an epidemic episode, the prevalence might increase up to as high as 90% (Teerakut et al. 1990).There is no published data on the prevalence of dengue infection among patients presenting with undifferentiated fever from Malaysia. Chin et. al. in 1993 reported 69.4% and 85.7% of clinically diagnosed dengue fever and dengue haemorrhagic fever respectively were serologically positive. However, these were among patients with clinically diagnosed dengue infection and not all patients with undifferentiated fever (Chin et al. 1993).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Non-dengue febrile thrombocytopaenia is a significant entity among adult patients with acute non-specific febrile illness. 19.5% of these patients had non-dengue febrile thrombocytopaenia and 40% of thrombocytopaenic febrile patients were seronegative for dengue infection. It may not be appropriate to manage them as dengue infection because the course of the illness may differ significantly from dengue infection. A future study on this group of patients is needed in order to gain better understanding of its aetiological agents, natural history and hence guide us on the appropriate management.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	We are grateful to Dr. Leelavathy, Dr. Hazizi, Dr. Aida, Dr. Wasilah, Dr. Nor Azila, M/A Azhar and Fairul for their participation in collection of data. I would like to thank all the laboratory assistants from Batu 9 Health Clinic and Primary Care Clinic Hospital Universiti Kebangsaan Malaysia Bandar Tasik Selatan, who helped with the laboratory tests</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(373, 370, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Breast cancer is the most common malignancy amongst women of all ages and is on the rise in Malaysia (Usha Devi, 2001). It is also the number one cause of death among Malaysian women. A breast cancer diagnosis for women is regarded with more fear than any other diagnosis, instilling fear of shock, grief, uncertainty and loss of control. Studies have indicated that breast cancer patients have extensive information needs and seek as much information as possible about the likelihood of cure and the disease process (Davison et al., 1995, Graydon et al., 1997).&nbsp; Women upon diagnosis of breast cancer want to know whether there is a cure for breast cancer. This important information need was found to be the top priority information for British and Canadian women (Luker et al., 1996). Women find this information important and of priority to deal with their illness (Chalmers &amp; Thompson, 1996).</p>\r\n<p>\r\n	Many women with breast cancer seek cure from different health bodies. Women here, seek all kinds of treatment to find a cure including &ldquo;witchcraft&rdquo;, traditional, herbal and Ayurvedic medicine till their dying day (Omar, 2000). Seeking help and cure from alternative health professional is a practice prevalent amongst Malaysian women, particularly as such treatment is non-invasive and does not lead to any blood loss or damage in their body image. Malay women still continue to choose the traditional method of seeking a &ldquo;witchcraft&rdquo;, whereas Chinese and Indian women choose herbal medicine from &ldquo;Sinseh&rdquo; and Ayurvedic medicine to seek cure for breast cancer. Women in Malaysia do not get enough information on cure. Women are ignorant of the fact that breast cancer survival rate is improved with early detection and early treatment. Seeking cure with unproven alternative treatment upon diagnosis of breast cancer has led to a low survival rate among women with breast cancer in Malaysia (Najibah, 2001). Irrespective of whether they were educated or uneducated, Malaysian women wanted more control over their health and sought information about the cure for breast cancer. Information on cure did not end only upon diagnosis but was found to continue even during their recovery phase.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Information on cure is a necessary prerequisite to informed decision making (Cawley et al, 1990). The information on cure could be the key to the process of empowerment. The current sources of information available on brochures and pamphlets do not reveal any information on cure, which could assist in their decision making.</p>\r\n<p>\r\n	Women upon diagnosis of breast cancer look upon nurses and doctors for information about the disease and for opinions. These women need to be provided with sufficient information on cure as a guide for decision-making.</p>\r\n<p>\r\n	Information about the likelihood of cure of breast cancer has been perceived as the most important information at diagnosis in a UK study (Luker et al, 1996). No study has been done locally to explore the importance of cure among women with breast cancer.</p>\r\n<p>\r\n	The aim of this study is to explore whether the information on cure was important to women newly diagnosed with breast cancer and to determine whether the variables such as age, level of education, social class and ethnic group had any influence on the information on cure.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHOD</strong></p>\r\n<p>\r\n	A survey design using a structured interview was adopted for this study. The study was conducted in Hospital Universiti Kebangsaan Malaysia (HUKM) and Hospital Kuala Lumpur (HKL). Approval to conduct the study was obtained from the Research Ethics Committee and from the directors of both hospitals. To examine the preference for information, nine information needs devised by Degner et al. (1997) were presented. Prior approval was obtained from Lesley Degner to use the nine information needs in this study.&nbsp; The nine information needs were presented in four languages: English, Malay, Mandarin &amp; Tamil and women were given the nine information needs according to their preferred language. Women were asked to choose which information is of importance and priority.</p>\r\n<p>\r\n	One hundred consecutive women newly diagnosed with breast cancer, between 3-4 months from diagnosis were interviewed. All the women in this study had undergone mastectomy and the interview was conducted in a private room. Informed consent was obtained prior to the interview. The sample comprised Malay (n=50), Chinese (n=30) and Indian (n=20). The patients&rsquo; age were categorised into two groups. The mean age of the study participants was 45 years (ranging from 32 to 60 years).&nbsp; Level of education was categorized as Standard 6, Form 3 (primary education), Form 5, Form 6 (secondary education) and graduate. Social class was allocated in accordance to the nature of work (occupation) of study participants and their husband. Professional groups were categorized into social class I, administrative and managerial workers into social class II, clerical and technicians into social class III, sales and related workers into social class IV, agricultural workers into social class V and lastly, factory workers, labourers and transport operators into social class VI(Table 1).</p>\r\n<p>\r\n	All data were coded using the data entry program of SPSS and details were recorded on demographic variables including age, level of education, ethnic group and social class. The Thurstone scaling program gave the scale values (score) for how many times each item was preferred. An Analysis of Variance (ANOVA) and t-test (t) was carried out to look at the differences between the mean scores.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Information on the likelihood ofcure obtained the highest importance with a score of 1.83 (Table 2). Information on sexual attractiveness achieved a score of 1.04 followed by spread of disease (0.86), different types of treatment (0.65) and side-effects of treatment (0.01). The other perceived important information needs were about the risk to family with a score of (-0.66), social life (-1.09) and self-care &nbsp;&nbsp;&nbsp;&nbsp; (-1.30). The information on impact on family and friends obtained the lowest score (-1.34). There was no significant difference in the scores for &ldquo;likelihood of cure&rdquo; between the various ethnic groups, educational level, social class, and age (Table 3, 4 &amp; 5).</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	The results of the present study suggest that the information on cure is the topmost priority information for women newly diagnosed with breast cancer, irrespective of age, level of education, social class and ethnic group. Information on cure was ranked the highest and most important information compared to other information needs. This finding is similar to the findings of Luker (1995), Degner (1997), Graydon (1997), and Galloway (1997), whereby cure was also an important and priority information need among women with newly diagnosed breast cancer.</p>\r\n<p>\r\n	All women wanted information on cure to deal with their illness (Chalmers Thompson, 1996). This explains why women with breast cancer seek cure with various kind of alternative medicine including &ldquo;witchcraft&rdquo; (Omar, 2000) and end up in late presentation (Usha Devi, 2001). Women fear breast cancer and mastec-tomy, and many lacked knowledge on whether there is a cure for breast cancer. Adequate information on cure upon diagnosis of breast cancer is therefore very important, as it will encourage women to seek cure with western medicine instead of traditional or alternative medicine.</p>\r\n<p>\r\n	Information on cure is important, as it will guide them in their decision making and thus improve greater outcome through greater adherence to treatment and thus increase their survival rate (Usha Devi, 2001). Information on cure is important as women need this information to maintain hope or to reassure themselves that their own perception and knowledge are valid. As Graydon (1997) points out that the importance placed on certain diseases may be a reflection of a high degree of perceived threat, actual gap in knowledge or lack of comprehension of the information provided. In conclusion, information about the likelihood of cure was the most important and topmost priority for women newly diagnosed with breast cancer irrespective of ethnic group, age, level of education and social class. The findings of this study will be useful to health care professionals when providing information to women newly diagnosed with breast cancer.</p>\r\n', 1),
(374, 371, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Fragile X syndrome is caused by repetitive trinucleotide repeats (CGG)<sup>n</sup>, in the first exon of the fragile X mental retardation-1 gene (FMR-1) of the X chromosome (Fu et al. 1991). When affected individuals have a large amplification, the DNA of the entire 5&rsquo; region of the gene becomes abnormally methylated. This is associated with methyl-lation of an upstream gene promoter region (a CpG island) which results in transcript-tional suppression of FMR-1, resulting in disease. Therefore, most affected males have no detectable FMR-1 mRNA, and it is believed to be the cause of the phenotype (Warren and Nelson 1994). At least three folate sensitive fragile sites, namely FRAX-E, FRAXD, and FRAXF that map to distal Xq are involved, but only FRAXA is asso-ciated with the fragile X syndrome (Parrish et al. 1994; Sheila et al. 2000). FRAXA is located on the chromosome X at position Xq27.3 (Jin and Warren 2000), near the end of the long arm. It is the most common cause of mental retardation. It is inherited as an X-linked dominant disorder with re-duced penetrance with an incidence of 15-20% of all X-linked mental retardation cases (Pergolizzi et al. 1992; Tarleton and Saul 1993; Warren and Nelson 1994).</p>\r\n<p>\r\n	The severity of the disease is correlated with the length of the CGG tract. Affected males and females exhibit a massive ex-pansion resulting in greater than 200 copies or may increase, up to 1000 repeats. Such expansions are called &lsquo;full mutations&rsquo;.&nbsp; Nonpenetrant male and female carriers exhibit premutation alleles between 60 and 200 copies (Sarah et al. 2003).</p>\r\n<p>\r\n	Male carriers transmit only unstable premutations while female premutation carriers can have carrier offsprings with premutation or affected children with full mutations. The phenotypic manifestations of fragile X syndrome are not specific. These may include variable degrees of mental retardation, prominence of the jaw and forehead, dysmorphic ears, high-arched palate, macrocephaly, macro-orchidism and hyperactivity. Males with fragile X syndrome usually have mental retardation and often exhibit characteristic physical features and behavior. Affected females exhibit a similar but usually less severe phenotype, likely the result of ran-dom X-inactivation patterns (Warren and Nelson 1994).&nbsp;</p>\r\n<p>\r\n	The usual diagnostic analysis for fragile X syndrome is based on the detection of a fragile site by cytogenetic analysis, fol-lowing specific cell culture conditions that modulate deoxynucleotide triphosphate levels. This however, has been difficult and sometimes inadequate for diagnosis (Rousseau et al. 1991), because other fragile sites such as FRAXE also exhibit the fragile site characteristics as FRAXA mutation (Warren and Nelson 1994). Molecular diagnosis of the CGG expansion mutation has been developed using Southern hybridization methods. However, the Southern hybridization tool is relatively expensive, time-consuming, laborious and requires large quantities of genomic DNA (Soon et al. 2001). Methods using PCR technique appears to be rapid and efficient for screening (Brown et al. 1993). It utilizes flanking of specific primers to amplify a fragment of DNA spanning the repeat region. In this study, the PCR analysis was verified using DNA obtained from a fragile X family, two cases with unrelated mental retardation, and a normal control. Thus, the aim of this study is to establish a rapid and simple PCR method as a screening tool for fragile X syndrome.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Samples</em></p>\r\n<p>\r\n	A total of six samples were analysed. One of the samples was taken from a known fragile X positive male patient (T1), who was referred to the Haematology Unit. In 1998. Blood samples were also collected from his mother (T2) and father (T3) for cytogenetic analysis. The other two samples (T4 and T5) were randomly selected, that is from two patients presenting with dysmorphic features and delay in development. The control (TC) sample was taken from a normal healthy female, who was cyto-genetically negative for the FRAXA gene at the fragile site. A detailed family history was obtained from all these cases.</p>\r\n<p>\r\n	<em>Cytogenetic Analysis</em></p>\r\n<p>\r\n	Peripheral blood lymphocytes were cul-tured according to standard laboratory pro-tocol for fragile X analysis. A combination of the two-culture system was used. These included lymphocyte culture (Fragile X cul-ture) under folic acid and thymidine defi-cient condition (media 199 with 5% bovine serum, antibiotics penicillin and strepto-mycin, and PHA)and the other was a syn-chronised lymphocyte culture (1640 media with 5% fetal bovine serum, antibiotics penicillin and streptomycin, PHA and thy-midine 0.1 M final concentration). All cul-tures were incubated at 37 &deg;C for 72 hours. The culture was harvested after adding 0.1 ml colcemid (10&micro;g/ml) for 7 minutes for the fragile X culture. 0.2ml (20&micro;g/ml) of col-cemid was added 1 hour before harvesting time for the synchronised culture. The cul-tures were treated with 0.075M potassium chloride (hypotonic solution) and fixed in 3:1 methanol acetic acid mixtures. Slides were aged and Leishman banding was carried out. At least 50 to 100 well spread, stained metaphases were scored to deter-mine the presence of the fragile site on the X chromosome. Fragile sites appear as non-stainning regions, chromatid gaps, or less frequently, as breaks near the terminal end of the long arm of the X chromosome. Banded metaphases from the synchro-nised culture were also analysed to exclude the presence of chromosomal aberrations. Karyotyping was carried out using an image analyser (CytoVision, Applied Imaging).</p>\r\n<p>\r\n	<em>DNA Extraction</em></p>\r\n<p>\r\n	DNA extraction was performed at the time of diagnosis. Samples were collected in ethylenediamine-tetraacetic acid (EDTA) tube and DNA was extracted from whole blood using the standard protocol of lysis; proteinase K digestion, followed by ex-traction with a combination of phenol and chloroform and precipitation with cold ethanol.&nbsp; The extracted DNA is aliquot and stored at -20&deg;C.</p>\r\n<p>\r\n	<em>PCR Amplification and Analysis</em></p>\r\n<p>\r\n	The archived DNA samples were subjected to a touch-down PCR analysis based on a slight modification of a previously des-cribed method (Tan et al. 2000). In this modified method, the enzyme AmpliTaq polymerase was replaced with the enzyme AmpliTaq (gold) polymerase. Amplification was carried out in a thermocycler in 30 &micro;l reactions containing approximately 300ng of DNA template, PCR buffer, 10 pmol each of primers g (forward primer) and f (reverse primer),&nbsp; 1.5 U of AmpliTaq (gold) polymerase (Perkin Elmer), 10% dimethyl sulphoxide (DMSO) and 200&micro;M each dNTP. dGTP was substituted with 7-deaza-2&rsquo; dGTP in a ratio of dGTP (1.2) : 7-deaza-dGTP (1.0) to amplify the mutated alleles.&nbsp; For the negative control, DNA template was replaced by RNase free water.</p>\r\n<p>\r\n	The reaction mixture went through a &lsquo;touch-down procedure&rsquo;; &nbsp;denaturation at 95&deg;C for 8 minutes, followed initially by 5 cycles of denaturation at 95&deg;C for 90 seconds, annealing at 70&deg;C to 66&deg;C (decreasing by 1&deg;C per cycle) for 60 seconds and elongation at 72&deg;C for 120 seconds. The reaction mixture was then subjected to 35 cycles of amplification at 95&deg;C for 90 seconds, 65&deg;C for 60 seconds, 72&deg;C for 120 seconds, and a final exten-sion of 10 minutes at 72&deg;C. The PCR products were analysed on 1.0% agarose gel containing ethidium bromide using the gel documentation system. The amplicon sizes were determined by using a 100bp standard DNA ladder. The presence of a band of the correct size approximately 522bp for the normal alleles excludes the presence of an expansion mutation at the FRAXA locus. An increased number of CGG repeats revealed no detectable band on the gel, indicating full mutation of the FRAXA gene in an affected individual.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Results of the cytogenetic analysis showed no chromosomal aberrations in five (T2, T3, T4, T5, TC) out of six samples&nbsp; studied (Table 1.0). One sample (T1) demon-strated presence of a fragile-site. The fra-gile site appears as a gap or break on the long arm of the X chromosome (Figure 1.0). All of the samples however, showed cytogenetically normal karyotypes using banded metaphases.</p>\r\n<p>\r\n	PCR analysis was carried out on all genomic DNA samples. The migrations of PCR products were analysed. As presented in figure 2.0, the presence of a 522bp band (for the normal allele) was observed in lanes 1, 3, 4, 5 and 6, thus excluding the presence of an expansion mutation at the FRAXA locus. The sample (T1) from the known case of fragile X syndrome (lane 2) showed absence of this PCR product, (figure 2.0) indicating that he may have the full mutation.</p>\r\n<p>\r\n	The family history of the known fragile-X positive patient (T1) revealed that both of his male siblings have characteristics of mental retardation and facial dysmorphic features of fragile X syndrome. The family of the other five cases showed no history of mental retardation among their siblings and parents.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Over the past few years, the diagnosis of fragile X syndrome was based on the presence of a folate-sensitive fragile site at Xq27.3 (FRAXA) induced in cell cultures under conditions of folate deprivation. In this study, cytogenetic analysis of the affected individual (T1) showed fragile sites at the distal end of the long arm of chromosome X (figure 1.0) when cultured under folate deficient conditions. However, cytogenetic analysis was unable to determine whether the breakage or gap resulted from expansion of the FRAXA gene or from other fragile sites genes. For instance, other fragile sites such as FRAXE (Xq28), that is located approximately 600 kilobases distal to FRAX, also exhibit fragile sites under folate deficient culture (Knight et al. 1993). This has led to difficulty in the interpretation of fragile sites using the conventional cytogenetic method. Cytogenetic analysis is less sensitive for detecting affected individuals. It also results in a high degree of variability between individuals and laboratories.</p>\r\n<p>\r\n	Our PCR analysis utilises flanking specific primers to amplify fragments of DNA spanning the repeat region. It amplifies the normal repeat size or those that are within the normal range, thus produces products of the expected size (figure 2.0).&nbsp; As expected, in the affected patient (T1), there is absence of the band, indicating full mutation of the FRAXA alleles, because the repeats are too large to be amplified (Soon et al. 2001; Wang et al. 1993). However, expansion involving other fragile site genes which demon-strated breakage under folate deficient conditions, will be amplified, and detectable bands will be observed on the electro-phoresis gel. Thus, the PCR analysis is specific for detection of the FRAXA full mutation. It is also able to differentiate it from expansion due to other fragile site genes.</p>\r\n<div>\r\n	<p>\r\n		The modified PCR analysis as outlined in this study is more sensitive and informative as a screening tool for detecting full mutation, compared to cytogenetic analysis. The method has its limitations in that it is unable to give an accurate size determination of full muta-tions and also to distinguish between nor-mal and fragile X premutation carrier indivi-duals. Affected patients showing absence of the expected band and females who show normal CGG allele should therefore be investigated further using Southern blotting. Although the Southern blot is the key diagnostic tool to detect the mutations of the fragile X syndrome, it is relatively laborious, time consuming, needs large quantities of DNA material, and is expen-sive. It is therefore not a suitable method for routine screening of fragile X syndrome.</p>\r\n</div>\r\n<p>\r\n	Studies of family pedigrees are im-portant because they help in excluding male transmission of fragile X and also to determine the carrier status. For instance, the family history of the affected male revealed that both of his male siblings also showed characteristics of fragile X syn-drome, namely mental retardation and facial dysmorphic features. However, cyto-genetic and PCR analyses were not carried out on the male siblings because they were residing abroad at the time of diagnosis. The paternal family history was not signifi-cant and revealed high intellectual capa-cities in all generations. The mother of the affected male was suspected to be an obli-gate fragile X carrier. Studies have shown that women who are pre-mutation carriers can have carrier offsprings with pre-muta-tions or affected children with full mutations (Warren and Nelson 1994; Soon et al. 2001; Pieretti et al. 1991).The chance that the allele will increase to full mutation depends on its size in the mother. All affected males and the majority of affected females inherit the mutation from their mother. The other two patients showed no family history of mental retardation.</p>\r\n<p>\r\n	PCR amplification of CGG repeats in the FRAXA fragment is difficult, and special conditions are required. The incorporation of 7-deaza-2&rsquo; dGTP to stabilise secondary DNA structures allows successful ampli-fication of the CG-rich region (Tan et al. 2000; Latimer and Lee 1991). However, PCR products with a high content of 7-deaza-2&rsquo; dGTP, do not stain efficiently with ethidium bromide (Innis 1990; Latimer and Lee 1991).&nbsp; Presumably, the presence of 7-deaza-2&rsquo; dGTP alters the electronic struc-ture of bound ethidium so that it can no longer fluoresce (Latimer and Lee 1991).</p>\r\n<p>\r\n	To investigate whether these changes may be an appropriate alternative to the described procedure, we have used the ratio 1.0: 1.2 respectively of 7-deaza-2&rsquo; dGTP to dGTP instead of 1: 1 ratio as previously described (Tan et al. 2000). We found that by changing the ratio of 7-deaza-2&rsquo; dGTP to dGTP , our PCR reaction was able to successfully amplify the FRAXA allele within the normal range. Together with efficient staining by ethidium bromide direct visualization of the PCR product under ultraviolet light was obtained. Omitting 7-deaza-2&rsquo; dGTP or lowering the ratio of 1.0 : 1.2 of 7-deaza-2&rsquo; dGTP to dGTP is not recommended be-cause it will increase secondary DNA structures in the PCR reaction. This may result in non-specific bands appearing during electrophoresis. The minor changes of using AmpliTaq Gold&reg;DNA polymerase rather than AmpliTaq polymerase as a chemical hot start enzyme has resulted in the increased specificity and product yield without biological contamination of the PCR reaction. This modification has led to increased sensitivity of the reaction, dimi-nishing the formation of non-specific pro-duct while allowing detection of low target copy number of the PCR product.&nbsp;</p>\r\n<p>\r\n	Interpretation of fragile X also poses some problems. The usual precautions for the prevention of contamination of the PCR product must be observed.Shadow or heteroduplex bands must be distinguished between true abnormal alleles when non-denaturing electrophoresis is used. Lane background and faint bands are other problems faced and these should be inter-preted with caution as smears of expanded fragments can be very faint.</p>\r\n<p>\r\n	The PCR analysis carried out in this study is a basic and simple technique that is completely independent of methylation and is specific for the FRAXA locus. Our study indicated that PCR analysis using specific primers was more sensitive than cytogenetic analysis to screen for full muta-tion of the fragile site. Southern blot ana-lysis should still be used for confirmation of the presence of an expanded allele. Typi-cally, molecular diagnostic services should include a combination of both Southern blot-based and PCR-based analysis to cover instances in which one or both types of tests are needed. G-banded karyotyping should be routinely performed as constitu-tional chromosome abnormalities have been identified as frequently as FMR-1 mutations in mentally retarded individuals referred for fragile X testing. Laboratories often need to use more than one method because no single method can detect all types of mutations equally well or with equal weakness (Maddalena et al. 2000).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, we have established a simple PCR analysis for screening fragile X syndrome. The PCR is an alternative method with advantages for mutational analysis. It is rapid, sensitive and uses minimal DNA. We recommend that labo-ratories use PCR as a screening tool for fragile X syndrome. Family history should also be evaluated to assist in the diag-nostic screening and to determine the risk of having affected children. Southern blot should also be carried out to detect repeat trinucleotide and methylation status for samples that have failed to amplify (male) or show a normal allele (females). In spite of the advances in molecular testing, rou-tine G-banded cytogenetic analysis should still be performed particularly for cases of unexplained learning difficulties and deve-lopmental delay to exclude other chromo-somal abnormalities.</p>\r\n<p align="left">\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	Authors would like to thank Dr Ng Kok Han, Director of the Institute for Medical Research Kuala Lumpur and Dr Lye Munn Sann, the former director, for their support and also Mr Ng Chyan Leong for his tech-nical advice. Our appreciation also extends to all staff of the Cytogenetics Laboratory, Molecular Pathology Unit and the Mole-cular Genetic Unit for their generous help, contribution and tremendous support to this study.</p>\r\n', 1),
(375, 372, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Microdeletion syndromes are genetic syndromes associated with small chromosome deletions which usually involved 1 to 3 million base pairs of DNA (Koerf&nbsp; 2002). Most of the syndromes is a contiguous gene syndrome;a disorder due to deletion of multiple gene loci that are adjacent to one another. The variation in the symptoms of the disease is related to the amount of genetic material lost in the chromosomal deletion. Almost all micro-deletion syndromes involved mental retardation or developmental delay and are usually associated with other abnormalities such as cardiac defects (Mathews 1999).</p>\r\n<p>\r\n	Some of the common microdeletion syndromes are Prader-Willi syndrome, Angelman syndrome, Williams syndrome, DiGeorge syndrome, Miller-Dieker syndrome, Kallmann syndrome and Smith-Magenis syndrome. Williams syndrome is the result of a commonly deleted region (approximately 1.5Mb) of 7q11.23 which contains the Elastin (ELN) gene locus (Ewart 1993, Korenberg et al 2000). Miller-Dieker syndrome involves a deletion of 17p13.3 locus which contains the lissencephaly gene (LIS1) (Ledbetter et al 1992). Prader-Willi syndrome is a contiguous gene disorder resulting from deletion of the 15q11-q13 region (Butler et al 1986).DiGeorge syndrome is caused by a large deletion of 22q13 (Greenberg et al 1988).Kallmann syndrome is an X-linked disorder associated with a microdeletion within Xp22.3 of chromosome X.</p>\r\n<p>\r\n	Many of these syndromes have long been recognized by medical geneticists, but have no further laboratory tests for confirmation. This is due to the fact that the microdeletions involved in these syndromes are generally beyond the resolution of routine conventional cytogenetics ie. karyotyping (Callan et al 1992).</p>\r\n<p>\r\n	The ability of fluorescence in situ hybridisation (FISH) to detect much smaller chromosomal abnormalities than can be detected with karyotyping has already been well documented (Ward et al 1999). FISH relies upon the presence or absence of a fluorescent signal to identify chromosomes rather than a specific banding pattern. It allows the surveillance of more cells and requires a much smaller sample than karyotyping.</p>\r\n<p>\r\n	This study was carried out to demonstrate the role of conventional cytogenetics (karyotyping) and FISH methods in the diagnosis of microdeletion syndromes.</p>\r\n<h4 align="left">\r\n	MATERIALS AND METHODS</h4>\r\n<p>\r\n	<br />\r\n	This is a retrospective study carried out by the Cytogenetics Unit, Hospital Universiti Kebangsaan Malaysia (HUKM). A total of nine cases of microdeletion syndromes were investigated from year 2002-2004. They include three cases of Prader-Willi syndrome, three cases of DiGeorge syndrome, one case of Miller-Dieker syndrome, one case of Williams syndrome and one case of Kallmann syndrome. Chromosomal analysis of each patient was carried out using both conventional and FISH methods.Chromosomes were prepared from peripheral blood lymphocyte cultures following standard procedures (Brown and Lawce 1997). GTG banding patterns (Gband chromosomes treated with trypsin and stained with Giemsa) of metaphases were analysed.&nbsp; A total of 20 metaphase spreads were analysed in each case. The karyotype was described in accordance with the International System for Human Cytogenetic Nomenclature (ISCN) (Mitelman 1995).</p>\r\n<p>\r\n	For FISH analysis, the locus specific probe kit (Vysis, USA) used are as listed:</p>\r\n<p>\r\n	(a)&nbsp; LSI Prader-Willi/Angelman region probe (D15S10) spectrum orange / CEP 15(D15Z1) spectrum green / PML spectrum orange and probe (SNRPN) spectrum orange/CEP 15 (D15Z1) spectrum green/PML spectrum orange;</p>\r\n<p>\r\n	(b)&nbsp; LSI DiGeorge N25(D22S75) region probe spectrum orange / ARSA (22q13.3) spectrum green control probe and TUPLE 1 spectrum orange / ARSA (22q13.3) spectrum green control probe;</p>\r\n<p>\r\n	(c) LSI LIS1 (Miller-Dieker) probe spectrum orange / RARA spectrum green.</p>\r\n<p>\r\n	(d)&nbsp; LSI Williams syndrome (elastin gene) region probe spectrum orange / D7S486, D7S522 spectrum green control probe; and</p>\r\n<p>\r\n	(e)&nbsp; LSI Kallmann (KAL) region probe spectrum orange / CEPX spectrum green control probe.</p>\r\n<p>\r\n	A minimum of 10 metaphases were analysed after selection for a positive signal at the control locus. Presence of microdeletion was indicated by absence of fluorescent&nbsp;signal&nbsp;of&nbsp;the&nbsp; specific&nbsp;locus&nbsp;on the respective chromosomes.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Of the total of nine cases studied, eight showed normal karyotype; five cases with 46,XX and three cases with 46,XY. FISH analysis done on the cases (cases 1 to 8) revealed deletion in the locus/loci responsible for the syndromes, therefore confirm the clinical diagnosis (table 1).</p>\r\n<p>\r\n	Case number 9 (DGS) showed translocation of the whole of the long arm of chromosome 22 onto the short arm of chromosome 9 (figure 1). Further analysis done on this case using FISH showed deletion on the locus responsible for DGS (figure 2).</p>\r\n<p>\r\n	Both of the parent&rsquo;s blood were also obtained and analysed to rule out familial DGS. The karyotype of the father showed normal chromosomal constitution (46,XY) whilst the mother&rsquo;s karyotype showed structural abnormality involving chromo-somes 9 and 22 (figure 3). FISH analysis using probe for DiGeorge (TUPLE1 and D22S75)showed no microdeletions involved in the mother; indicating that she is a carrier.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Cytogenetic techniques have improved over the years and have made it possible for microdeletion syndromes to be described. Most of the microdeletionsyndromes involve multiple genes and can be considered as contiguous gene deletion syndromes e.g. William syndrome in which the deletion involves the elastin gene and a protein kinase LIMK1, and Prader-Willi and Angelman syndromes which involved many genes in the 15q11-q13 region (Korenberg et al 2000,&nbsp; Butler 1986).</p>\r\n<p>\r\n	In the present study, all of the cases showed normal karyotype by conventional cytogenetic analysis, except for one case, which showed structural chromosomal abnormality involving chromosomes 9 and 22 (case 9). However, microdeletion was revealed in all of the cases studied by FISH analysis. The reason for the discrepancy in the results between conventional and FISH analysis seen in this study is that the latter method is able to detect deletions at a greater resolution. In a study done on Prader-Willi and Angelman syndromes, it has been reported that even high-resolution cytogenetics (a resolution higher than 550 bands) cannot detect the deletions involved (Smith et al 1995).</p>\r\n<p>\r\n	The application of FISH technique in the diagnosis of microdeletion syndromes has been shown to be extremely useful in many previous studies (Smith et al 1995, Demetrick 1999)as well as in the present study. FISH reveals microdeletion in the locus/loci responsible for the syndromes in eight cases (case 1-8) studied which otherwise shows normal karyotype. However, due to some cases of microdeletion syndromes which are caused by mechanisms other than chromosome deletion, conventional cytogenetics should always be applied along with FISH. As clearly demonstrated in case 9 from our study, the chromosomal abnormality was detected by the conventional method, while FISH identified microdeletion in the region involved with DiGeorge syndrome ie. 22q11.2. Another study has shown that about 5% of Prader-Willi syndrome and Angelman syndrome involved rearrangements of chromosome 15. For this reason, FISH cannot be the sole technique without conventional cytogenetics (Donaldson et al 1994).</p>\r\n<p>\r\n	Approximately 90%-96% of DGS patients show a microdeletion of the 22q11.2 region (Driscoll et al 1992). Two of three patients with DGS in the present study have normal karyotypes by chromosome analysis, except in one case (case 9) where a deletion in the 22q11.2 region was suspected. By using FISH technique with DiGeorge critical region probe, all three patients were confirmed as having a 22q11.2 deletion, thus establishing DGS in these patients (100% concordance).</p>\r\n<p>\r\n	In 70% of PWS patients, an interstitial deletion of the (15)(q11q13) region derived from the paternal chromosome is a characteristic feature (Ledbetter et al. 1981).About 30% of PWS cases are due to maternal uniparental disomy (Nicholls&nbsp; 1993). FISH analysis with PWS region probe confirmed the deletion in all of the three cases in this study (table 1).</p>\r\n<p>\r\n	The phenotype of patients with Williams syndrome (WS) becomes variable with advancing age which makes diagnosis more challenging. However, since complete deletion of one elastin allele on chromosome 7 at band 7q11.23 in WS patients has been demonstrated, diagnosis is now easier. While chromosome analysis showed normal karyotype in our single case of WS, a deletion of elastin locus was identified by FISH analysis.</p>\r\n<p>\r\n	There is one case of Miller-Dieker syndrome (MDS) and another of Kallman syndrome which were also confirmed by FISH in this study. The main indicators of MDS are severe brain damage with microcephaly and severe mental as well as psychomotor retardation. The gene responsible for the X-linked form of Kallmann syndrome, KAL1, encodes a protein, anosmin, which plays a key role in the migration of gonadotropin-releasing hormone (GnRH) neurons and olfactory nerves to the hypothalamus. Although mental or intellectual disturbance was described (Kallmann et al 1944), analyses of the genotype-phenotype relationship showed that Kallmann syndrome patients with mental disorders have large deletions on Xp22.3 that extends beyond the KAL1 locus (Nagata et al 2000). In contrast, almost all patients with mutations restricted to the KAL1 locus are free of mental disturbance.</p>\r\n<p>\r\n	Although the microdeletion syndromes mentioned above have distinctive clinical features, they tend to have in common the characteristics that they are usually sporadic but occasionally are transmitted in families as dominant traits. Most of the microdeletions occur spontaneously but individuals who have a deletion, if they are able to have children, have a 50% chance of transmitting the deletion to any child. In the absence of a parental deletion, there is a low risk of recurrence as germ line mosaicism is rare.</p>\r\n<p>\r\n	In view of the structural chromosomal abnormality from case 9, the findings from both the conventional and FISH analysis suggest that the patient has a familial DiGeorge syndrome; the syndrome has been transmitted from the mother. Genetic counseling was advised for the couple since they have a high risk of having another abnormal child and recurrent abortions.</p>\r\n<p>\r\n	The fact that FISH is a more powerful method compared to conventional karyotyping has been demonstrated in this study as well as other studies (Ward et al 1999, Demetrick 1999). Besides being rapid, sensitive and simple, FISH technique can also be performed on interphase cells hence interphase FISH was introduced.&nbsp; It can be applied for haematological malignancies, for example in the identification of <em>bcr/abl</em> gene fusion in chronic myeloid leukemia (CML) and prenatal test for detection of common trisomies of chromosomes 13,18,21, as well as X and Y chromosomes (Cianciulli et al&nbsp; <strong>2004,</strong>Hogge et al 1996).&nbsp;</p>\r\n<p>\r\n	Despite all the advantages mentioned above, there are two disadvantages of FISH; it will only provide information about the probe being tested, other aberrations will not be detected and the probes used are relatively expensive. However, due to the increased accuracy and time saved compared with the more laborious high-resolution cytogenetic analysis, FISH may be more cost effective than the conventional method for detection of deletions eventhough the probes are relatively expensive.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, the present study has proven that combined FISH and conventional cytogenetics complement each other in the diagnosis of microdeletion syndromes. It is important for confirmation of the diagnosis, genetic counseling, and facilitation of prenatal testing.</p>\r\n<h4 align="left">\r\n	ACKNOWLEDGEMENTS</h4>\r\n<h4 align="left">\r\n	&nbsp;</h4>\r\n<p>\r\n	We would like to thank Norul Shima Ali and Julia Mohd Idris from the Cytogenetic Unit, Hospital UKM for their technical support.</p>\r\n', 1),
(376, 373, '<div>\r\n	<p>\r\n		<strong>Introduction</strong></p>\r\n</div>\r\n<p>\r\n	Biphenotypic acute leukaemia (BAL) is a rare disease, which occurs in 5 to 10% of acute leukaemias. BAL can arise de novo or it may be secondary to previous cytotoxic therapy. As with other types of acute leukaemia, patients with BAL usually present with symptoms resulting from cytopenias. The blast count at diagnosis does not differ from that in acute myeloid leukemia (AML) or acute lymphoid leukaemia (ALL). BAL can present at any age, including children, although it is more common in adults. The blasts cell morphology are variable; some cells may display myeloid differentiating features such as azurophilic granules or Auer rods; others may have lymphoid or undifferentiated morphology. In some cases, two blast populations may be seen, consisting of a population resembling myeloblast with another population of smaller lymphoid-looking blasts. Immunophenotyping is mandatory to establish the diagnosis of BAL. These blasts co-express myeloid and T or B lineage specific antigens. Diagnosis is based on a scoring system adopted by the European Group of Immunological Classification of Leukaemias (EGIL) (Bene et al 1995), and it has been included in the WHO classification of haemopoietic malignancies as acute leukaemia of ambiguous lineage (Brunning et al 2001). There is no specific cytogenetic abnormality in BAL. Studies done by Carbonell F et al (1996) had shown that the most common cytogenetic abnormalities were t(9;22)(q34;q11) and abnormalities of chromosome 11, particularly 11q23. BAL is very difficult to treat with many cases being resistant to induction chemotherapy; and those which achieved morphological remission had a higher risk of relapse. The prognosis of BAL in adults is worse than AML or ALL and most are associated with over-expression of P glycoprotein (Pgp)(Legrand et al 1993).</p>\r\n<p>\r\n	<strong>Case Report</strong></p>\r\n<p>\r\n	<em>Case 1</em></p>\r\n<p>\r\n	AS, a 45-year-old policewoman was admitted to HUKM with severe back pain of one day.&nbsp; She also gave a history of loss of appetite and loss of weight of two months duration. She was diagnosed to have hypertension, ischaemic heart disease and hyperlipidaemia. On physical examination, she was pale, with enlarged liver and spleen. No lymph node enlargement or hypertrophy of the gums was observed. No bleeding tendency was noted. Vital signs were stable. Examination of other systems were normal. Laboratory investigation and results are shown in Table 1.</p>\r\n<p>\r\n	The patient was given induction chemotherapy consisting of cytosine arabinoside for seven days and daunurobicin for three days. Bone marrow assessment at day 28 post chemotherapy showed that she has achieved marrow remission. She was given the same regime for her first consolidation treatment and for her second and third consolidation; the chemotherapy regimen was changed to mitoxantrone for two days and cytosine arabinoside for four days. Throughout the post chemotherapy period, she developed severe myelosuppression with neutropenic sepsis, thrombocytopenia and anaemia. She was planned for allogeneic peripheral blood stem cell transplantation but while waiting for the work out, she had a relapse. She was then given salvage chemotherapy which consists of Fludarabine, cytosine arabinoside and idarubicin (FLAG-IDA) with growth factor support.&nbsp; Unfortunately, 10 days post chemotherapy, she developed severe neutropenic sepsis with shock and she succumbed to her death.</p>\r\n<p>\r\n	<em>Case 2</em></p>\r\n<p>\r\n	RO, a 30-year-old Malay lady was referred to HUKM from Putrajaya Hospital with a history of fever of two weeks duration, blurring of vision of her left eye, fatigue and lethargy.&nbsp; On admission, she was very pale. There were multiple ulcers over her lips and palate, and she had gum hypertrophy. Multiple bilateral cervical lymphadenopathy were also noted. Vital signs were stable. Fundus examination showed presence of left retinal haemorrhages with dilated vessels. Cardiovascular and respiratory systems were normal. Abdominal examination showed hepatosplenomegaly measuring 4 cm and 3 cm below the costal margins respectively.Laboratory investigations and results are shown in Table 1.</p>\r\n<p>\r\n	This patient was also given induction chemotherapy consisting of cytosine arabinoside for seven days and daunorubicin for three days. In the post chemotherapy period, the patient developed persistent pancytopenia, complicated with neutropenic sepsis. A month after chemotherapy, her peripheral blood showed presence of residual blasts. Bone marrow aspiration confirmed the persistence of disease. A second induction chemotherapy was started with a regimen consisting of cyclophosphamide, dexamethasone, doxorubicin and vincristine. However, during chemotherapy, she developed severe upper gastro-intestinal bleeding and succumbed to death.</p>\r\n<p>\r\n	<strong>Discussion</strong></p>\r\n<p>\r\n	Biphenotypic acute leukaemia (BAL) is a form of acute leukaemia of ambiguous lineage.&nbsp; It is a rare entity which is difficult to define previously, due to lack of consistent diagnostic criteria. The characterisation of these disease based on morphology alone can be difficult. Blasts are often undifferentiated, with no distinguishing characteristics. However, some cases have been described based on the detection of two distinct populations of blasts which vary in size. Our two cases showed two distinct populations of blasts morphologically. The larger blast cells with moderate to abundant cytoplasm are positive for peroxidase stain and the smaller blast cells showing scanty cytoplasm, are negative for peroxidase stain.&nbsp;</p>\r\n<p>\r\n	Immunophenotyping is the most important diagnostic tool in characterizing BAL. Diagnostic criteria have been defined including those outlined by EGIL(Bene, M.C. et al 1995) and WHO (Brunning RD et al 2001).&nbsp; The diagnosis is based on a scoring system for markers proposed by the EGIL, which defines the minimum number of phenotypic characteristics necessary to be considered true biphenotypia. Patients who do not meet these criteria will be diagnosed as either AML with lymphoid aberrant markers or ALL with myeloid aberrant markers. Markers that have been used and considered to be most specific to differentiate the three cell lines are shown in Table 2. Each marker has a different score. The score encompasses the number and degree of specificity of the markers expressed by the leukaemic cells. The diagnostic criteria are (i) there should be at least a score of 2 from each lineage, and (ii) both myeloid and lymphoid markers are expressed on the same cells in order to diagnose BAL. This is to differentiate biphenotypic acute leukaemia from bilineage acute leukaemia.&nbsp; In these two cases, the blast cells expressed intra CD22, and intra MPO which fulfilled criteria (i) and the co-expression of CD 19 and CD 33, CD 13 and CD 10 in both cases fulfilling criteria (ii) which confirmed the diagnosis of BAL. In contrast, bilineage acute leukaemia has two distinct population of leukaemic cells. The larger cells will express myeloid markers while the smaller cells will express B or T-lymphoid markers. Previous studies of BAL have suggested that these leukaemias may arise by malignant transformation of a pluripotent stem cell. The finding of a significantly greater percentage of biphenotypic cases with CD34 expression compared to non-biphenotypic cases in a study done by Legrand O et al (1998), supports the concept that this transformation process occurs at a step close to the haemopoietic progenitor cell. In the same study, the percentage of BAL patients with unfavourable karyotype was higher compared to AML (60% versus 20%). The high percentage of unfavourable karyotypes especially abnormalities of chromosome 11q23 and t(9;22) has been shown by a few studies (Carbonell et al 1996, Hanson et al 1993). One of the patients had an unfavourable karyotype, the Philadelphia chromosome, and molecular analysis also showed presence of BCR-ABL gene. The study by Legrand O et (1998) al also showed that most BAL patients have P-glycoprotein (Pgp) overexpression which correlates with poor treatment outcome. Treatment outcome of BAL with standard AML protocol has been proven to be unfavourable and yet there are no standard induction protocol for BAL; even those patients who attained remission has a higher risk of relapse. The majority of the patients received chemotherapy for AML and once the patients attained morphological remission, upfront stem cell transplantation will offer a better outcome. Both of our patients received the AML induction regime; patient 1 went into marrow remission and was planned for allogenic peripheral blood stem cell transplantation. Unfortunately, there was no matched sibling donor available, and she relapsed about 10 weeks after the fourth course of chemotherapy. The second patient did not achieve marrow remission. Legrand O et (1998) al have documented that the outcomes of treatment in patient with BAL are worst when compared to AML or ALL. The causes of poor prognosis in patients with acute BAL appear to be related to intrinsic cell characteristics (increase of unfavourable karyotypes, overexpression of Pgp and immature cell markers). Therefore, more studies on the induction chemotherapy protocols for this disease is important not only to improve the treatment outcome but also to minimise the severe adverse effects of the treatment protocols.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(377, 374, '<div>\r\n	<p>\r\n		<strong>Introduction</strong></p>\r\n	<p>\r\n		Labyrinthitis ossificans is a pathologic ossification in the cochlea and labyrinth(Aferzon M. &amp; Reams C.L. 2001). It is usually caused by an infection that reaches the inner ear via either the tympanogenic (most common), meningogenic or hematogenic route. Bacterial meningitis is the most common cause of meningogenic labyrinthitis. The infection spreads to the inner ear via subarachnoid spaces such as the cochlear aqueduct and the internal auditory canal. Infection in the subarachnoid spaces (meningitis) also can reach the cochlea via hematogenous spread. Ossification secondary to meningitis is typically bilateral. In the early years of cochlear implant surgery, ossification of the cochlea was a contraindication for cochlear implant surgery. As surgeons gained more experience with implantation, they found that some degree of ossification could be dealt with through modifications of the surgical technique.</p>\r\n	<p>\r\n		<strong>Case reports</strong></p>\r\n	<p>\r\n		<em>Case 1</em></p>\r\n	<p>\r\n		&nbsp;MF, a 14-year-old Malay boy was diagnosed to have postlingual sensorineural hearing loss secondary to head injury.&nbsp; He was involved in a motor vehicle accident (MVA) at the age of 11 years. The high resolution computed tomography (HRCT) and magnetic resonance imaging (MRI) of the temporal bone showed labyrinthitis ossificans. He underwent uneventful surgery without any complications one and half years after the MVA.&nbsp; Intraoperatively there was moderate ossification in the scala tympani.&nbsp; An extended cochleostomy was performed until an opening was found deep at the basal turn of the cochlea.&nbsp; A straight electrode was inserted.&nbsp; However, only partial insertion was achieved up to 11 electrodes.</p>\r\n	<p>\r\n		<em>Case 2<span>&nbsp;</span></em></p>\r\n	<p>\r\n		<span>&nbsp;</span>DLKM, a 4-year-old Chinese boy had normal hearing in both ear before he had <u>Haemophilus</u> <u>influenzae</u> meningitis at the age of three years 4 months. He was treated with intravenous antibiotics in Malacca General Hospital for about 3 weeks. He developed epilepsy for which he was treated with Syrup Sodium Valpoorate 375mg bd. Clinical examination was unremarkable and his high resolution computed tomography (Fig.1) and magnetic resonance imaging of the temporal bone showed features of labyrinthitis ossificans (Fig. 2, 3). An extended cochleostomy was initially performed but there was no opening found.&nbsp; A 2 mm tunnel was drilled at the basal turn anteriorly.&nbsp; A straight electrode was partially inserted.&nbsp; Seven months after the operation, he could respond to loud sounds and was able to say simple words such as &ldquo;papa&rdquo; and &ldquo;mama&rdquo;.<span>&nbsp;</span></p>\r\n	<p>\r\n		<em>Case 3</em></p>\r\n	<p>\r\n		LWT, a 6-year-old Chinese boy with profound sensory neural hearing loss (SNHL) secondary to meningococcal meningitis at the age of 5 years, was diagnosed to have profound SNHL. About 5 days after being discharged from the hospital, the mother noticed that he did not respond to sound. A hearing aid was tried for 6 months but with no benefit.&nbsp; His HRCT of the temporal bone was normal.&nbsp; MRI was not performed. However, intraoperatively there was fibrous tissue in the scala tympani noted during cochleostomy.&nbsp; No ossification was found.&nbsp; A straight electrode was inserted through the cochleostomy.&nbsp; Only partial insertion was achieved (Fig. 4). There was no postoperative complications.</p>\r\n	<p>\r\n		<strong>Discussion</strong></p>\r\n	<p>\r\n		Hearing loss following an episode of bacterial meningitis is a well-documented phenomenon with incidence ranging from 5% to 35%(Kim HY et. al. 2001). Fortnum and Davis (1993) reported that up to 10% of children who suffer acute bacterial meningitis would sustain some degree of permanent unilateral or bilateral deafness. Weissman and Kamerer (1993) reported that approximately 2 to 5% of children and adults who survive a bacterial meningitis develop profound bilateral SNHL. The site is almost always at the cochlea where there is loss of the organs of Corti and not at the auditory nerve as was</p>\r\n	<p>\r\n		previously reported (Dodds A et. al. 1997). The infective process in the cochlea and labyrinth may produce a reaction in the endosteum resulting in new bone formation (Novak MA et. al. 1990).&nbsp; Post-meningitis SNHL can occur as early as 48 hours post infection.</p>\r\n	<p>\r\n		As early as two weeks after the onset of meningitis, fibrosis is present followed by new bone formation within two months (Niparko JK &amp; Mabrie DC. 1999). Ossification initially develops in the basal turn of the cochlea at the region of the scala tympani near the round window (Desouza C et. al 1991, Dodds A et. al. 1997).Radiographic evidence of ossification can be found as early as two months after the acute infection indicating that the intra cochlear process probably begins much earlier (Novak MA et. al. 1990).</p>\r\n	<p>\r\n		Green et al (1991) reported that a partially obliterated lumen can appear to be patent in the temporal bone scan as seen in case 3.&nbsp; A labyrinth that has undergone fibrous obliteration may appear normal on CT but will lack the high signal on T<sub>2</sub> weighted MRI (Harnsberger HR et. al. 1987, Weissman JL &amp; Kamerer DB. 1993). Therefore, it is advocated that MRI of the temporal bone is required in all post meningitis cases for cochlear implantation although they have normal HRCT.</p>\r\n	<p>\r\n		Balkany et al (1988) reported 15 patients with partially ossified cochlea where the results of cochlear implant were similar to those achieved in patients who had normal cochlea. Electrodes were completely inserted in 14 patients and partially in one patient (Balkany T et. al. 1988). Becker et al (1984) reported that mild labyrinthitis ossification is not a contraindication to cochlear implantation.</p>\r\n	<p>\r\n		Novak et al (1990) found that timing of implantation may be critical. A delay of even a few months probably would have allowed ossification to proceed to a degree that standard implantation procedures would have been impossible. Therefore the potential benefit of cochlear implantation may be reduced (Dodds A et. al. 1997, Novak MA et. al. 1990). In case 2 the operation was performed about 8 months post meningitis after discussing with the parents regarding the benefit of cochlear implantation. In case 3, the patient underwent cochlear implantation 11 months post meningitis.</p>\r\n	<p>\r\n		<em>Surgical technique in ossified cochlear</em></p>\r\n	<p>\r\n		Various surgical techniques have been proposed to minimize complications of electrode insertion in the ossified cochlea. Radical cochleostomy has been performed when extensive ossified cochlear is encountered. Gantz et al (1998) described a method of drilling out the entire basal turn of the cochlea to facilitate cochlear implantation in a completely ossified cochlea. Several authors have since reported satisfactory results in patients using the Gantz (1998) procedure (Weissman JL &amp; Kamerer DB. 1993, Rauch SD et. al. 1997, Lambert PR et. al. 1991). Limited tunnel techniques are more frequently used. These techniques are simpler, less time consuming and involve less potential for complication than extensive cochleostomy (Trinh BA et. al. 2000). However, these techniques can impose serious limitations to multichannel stimulation because of the high chance of partial insertion. Scala vestibuli insertions have also been used in the presence of a complete obstruction of the scala tympani.</p>\r\n	<p>\r\n		Steenerson and Garry (1999) described three patients with scala tympani ossification with open scala vestibuli and successful implantation in the scala vestibuli. Steenerson and Garry (1994) reported that scala vestibuli implantation is an alternative when ossification is found in scala tympani. They reported that scala vestibuli cross section area may be greater than scala tympani resulting in easier cochlear implant electrode insertion.</p>\r\n	<p>\r\n		<strong>Conclusion</strong></p>\r\n	<p>\r\n		It is advocated that all patients with meningitis undergo a detailed assessment of the cochlea with MRI despite a normal looking HRCT.&nbsp; Cochlear implantation in labyrinthitis ossificans is still feasible. However it should be performed by an experienced otologist who would be able to vary the surgical technique for electrode insertion according to severity of the ossification of the cochlea.</p>\r\n	<p>\r\n		<strong>Acknowledgements</strong></p>\r\n	<p>\r\n		We would like to thank our audiologists, radiographer, speech therapists and Mr Kamarulzaman Othman for their contributions.</p>\r\n</div>\r\n', 1),
(378, 375, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n</div>\r\n<p>\r\n	Juvenile laryngeal papillomatosis are benign epithelial neoplasms of the laryngeal mucosa, often multiple and recurrent with a tendency to spread throughout the respiratory tract (Moore &amp; Lattes, 1959).&nbsp; The most common age of onset is during the first 5 years of life. The lesions may regress spontaneously at puberty but occasionally the patients will have recurrences throughout life. Juvenile multiple laryngeal papillomas are known to be caused by human papillomavirus (HPV) infection especially HPV-6 and HPV-11 (Corbitt et al, 1988).</p>\r\n<p>\r\n	P53 is the protein product of a tumour suppressor gene on the short arm of chromosome 17 (Isobe et al, 1986). It is involved in the regulation of the cell cycle and the triggering of apoptosis. Mutations of this tumour suppressor gene are amongst the commonest detected in human malignancies (Lane, 1994).While the wild-type (normal) p53 is not usually detected by immunohistochemistry as it has a short half-life (6-20 min), most genetic alterations result in the accumulation of a stabilised protein in the nucleus which is readily detectable by immunohistochemistry in paraffin-embedded tissue (Levine, 1992; Wynford-Thomas, 1992). Positivity indicates that the normal function of p53 in directing apoptosis of genetically abnormal cells is no longer operating in the cell.</p>\r\n<p>\r\n	Oncoprotein c-erbB-2 (also known as <em>neu</em>/HER2) belongs to the family of the epidermal growth factor receptors, together with the Epidermal Growth Factor receptor (<em>c-erb </em>B1), Her3 (<em>c-erb</em> B3) and Her4 (<em>c-erb</em> B4), possesses tyrosine kinase activity, and is able to confer growth advantages to tumour cells overexpressing the protein and/or manifesting amplification of the cognate gene (Kurebayashi, 2001). <em>c-erb</em> B2 is a 185-kDa glycoprotein, which has been mapped to on chromosome 17q21 (Akiyama et al, 1986). Amplification of the gene has been demonstrated in adenocarcinomas arising at a number of different sites as breast, ovary and gastrointestinal tract (Tal et al, 1988).&nbsp;</p>\r\n<p>\r\n	Here we report two cases of laryngeal papillomas containing viral genomes of HPV by in situ hybridisation and p53 with &nbsp;c-erbB-2 protein expressions by immuno-histochemical method.</p>\r\n<p>\r\n	<strong>CASE REPORTS</strong></p>\r\n<p>\r\n	<em>Case 1</em></p>\r\n<p>\r\n	In 1986, a one-year-old Chinese male baby lost his voice for three months duration, but he responded to call. There was no productive cough, fever or congenital abnormality. Before this episode he was crying and there was no significant illness of note. He was a full-term baby and he was delivered by spontaneous vaginal delivery.</p>\r\n<p>\r\n	Examination under general anaesthesia showed laryngeal papillomatosis. Since then he had more than 40 direct laryngoscopes and tracheoscopy in an attempt to eradicate the papillomas. In 1987, a tracheostomy was performed.</p>\r\n<p>\r\n	Interferon therapy for a duration of one month was started in 1988 but this failed to eradicate the disease. In 1991, the papilloma had spread to the stoma, subglottic area and the upper part of the trachea.</p>\r\n<p>\r\n	Presently he is undergoing repeated direct laryngoscopy, tracheoscopy and plucking of the papillomas monthly. This is because the papillomas reappear after each surgical procedure. The latest biopsy was done in year 2001, and in 2002, he died of widespread involvement of the papillomas in the lungs.</p>\r\n<p>\r\n	<em>Case 2</em></p>\r\n<p>\r\n	A six years old Chinese girl from Johore presented to the Ear, Nose and Throat (ENT) clinic of Universiti Kebangsaan Malaysia (UKM) in 1993 complaining of hoarseness of the voice for the past one year. The hoarseness was gradual in onset but progressively increased in intensity. It was accompanied by productive cough with greenish sputum. There was no stridor, dyspnea or choking.</p>\r\n<p>\r\n	Initially, she sought treatment at another hospital and a direct laryngoscopy was done. A diagnosis of left vocal cord papilloma was confirmed histologically. She was referred for further management in UKM.</p>\r\n<p>\r\n	Examination under anesthesia, multiple laryngeal papillomatosis were detected arising from the left vocal cord, posterior half of left false cord and anterior commisure.</p>\r\n<p>\r\n	Further biopsies and laser vaporization were done. Later, she was subjected to another laser vaporization. The papillo-matosis had recurred in the same area and extended to one-third of the right vocal cord. There was no subglottic extension.</p>\r\n<p>\r\n	Due to the persistent nature of this condition, she underwent further exami-nation under anesthesia and repeated treatment. She was however lost to follow-up.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Both specimens consisted of multiple small fragmented laryngeal tissues, each measuring 4 x 3 cm and 1.5 x 1.5 x 1.5 cm in aggregate.</p>\r\n<p>\r\n	The specimens were fixed in 10% buffered formalin and sampled in accordance with the departmental manual. Light microscopic slides were prepared from these tissues fixed in formalin, embedded in paraffin, cut at 4&micro;m in thickness and stained with haematoxylin and eosin according to standard procedure.</p>\r\n<p>\r\n	Laryngeal papillomatosis biopsy specimens at the time of diagnosis (the above specimens) and from the last subsequent procedure (done in 2001) were used for detection of both p53 and c-erbB-2 proteins.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<em>Detection of p53 and c-erbB-2 proteins by immunohistochemical staining</em></p>\r\n<p>\r\n	Immunohistochemistry was performed on paraffin-embedded tissue using the labelled streptavidin biotin method (LSAB+kit, DAKO K 0690, Dako-Cytomation, Glostrup, Denmark). The mouse monoclonal antibody against p53 (DO-7, Dakopatts, at a dilution of 1:50) was used while polyclonal antibody against &nbsp;&nbsp;&nbsp; c-erbB-2 (DAKO A 0485, 1:300) was employed.</p>\r\n<p>\r\n	Briefly, 3 &micro;m sections were cut from the formalin-fixed, paraffin-embedded tissue blocks and mounted on silanized glass slides, deparaffinised, and placed in target retrieval solution (DAKO S1699, Dako-Cytomation, Glostrup, Denmark) and in a microwave 600W for 10 minutes. The slides were left to cool in room temperature for 20 minutes and then washed again. Subsequently, the slides were placed in 3% hydrogen peroxide for 5 minutes at room temperature. The tissue sections were then incubated with anti-Human p53 mouse monoclonal antibody (DO-7, 1:50) at room temperature for 30 minutes.</p>\r\n<p>\r\n	After several washes with Tris buffered saline (TBS), sufficient amount of biotinylated linking antibody was applied and the sections were incubated at room temperature for 15 minutes followed by washing in three changes of TBS. Labelled Streptavidin Biotin (LSAB, Dako-Cytomation, Glostrup, Denmark) was then applied and the slides were again incubated at room temperature for 15 minutes and subsequently washed with three changes of TBS.</p>\r\n<p>\r\n	Localisation of p53 was visualised by incubating the tissue sections for 10 minutes in substrate-chromogen solution (Dako Corp, Carpenteria, California). The slides were finally washed under running tap water, counterstained with haematoxylin, dehydrated, cleared and mounted. The positive control was from a known p53 positive tissue section of an infiltrating ductal carcinoma, and the negative control was from the tissue section of the same case but without primary antibody.</p>\r\n<p>\r\n	The reactivity for p53 protein was regarded as positive if distinct nuclear staining was present. Staining pattern was scored semi-quantitatively as follows: 0, no staining; 1, staining of the basal cells and suprabasal region up to one third of the epithelial thickness; 2, staining of two thirds of the epithelial thickness; and 3, staining throughout the epithelial thickness.</p>\r\n<p>\r\n	Staining for c-erbB-2 was performed as described for p53 with minor modifications.&nbsp; Heating in the microwave (600W) for 20 minutes was carried out followed by incubation with a 1:300 dilution of polyclonal rabbit anti-Human c-erbB-2 (DAKO A 0485, DakoCytomation, Glostrup, Denmark) at room temperature for 30 minutes.</p>\r\n<p>\r\n	The positive control was taken from a known c-erbB-2 positive section of an infiltrating ductal carcinoma and the negative control was obtained from the tissue section of the same case as above but without primary antibody.</p>\r\n<p>\r\n	c-erbB-2 positivity is scored according to: 0 = no membranous staining; 1+ = incomplete membranous staining; 2+ = membranous staining of moderate intensity in more than 10% neoplastic cells; 3+ = strong and complete membranous staining in more than 10% neoplastic cells.</p>\r\n<p>\r\n	<em>In situ hybridisation</em></p>\r\n<p>\r\n	In situ hybridisation was performed using human papillomavirus DNA probes commercially obtained from Dako (Carpenteria, CA, USA) and In situ Hybridisation detection System (DAKO<sup>&reg;</sup> in situ hybridisation detection system, Dako, Carpenteria, CA, USA), as described previously (12). The Wide Spectrum and the type specific HPV biotinylated DNA probes were prepared from HPV DNAs which were cloned into plasmid vectors. The targets for Wide Spectrum probe were the genomic DNAs of HPV types 6, 11, 16, 18, 30, 31, 33, 35, 45, 51 and 52. The specific HPV probes used were types 6, 11, 16, 18, 31 and 33. The positive control probe was prepared from total human DNA. The target was total genomic human DNA. The negative control was a plasmid DNA.</p>\r\n<p>\r\n	Positive reactions for hybridisation were shown as blackish purple stain mainly in the nuclei of infected cells containing target DNA and negative reactions of the epithelial cells were stained as light pink. Typing with Type-Specific HPV probes was done on specimens showing positive signals with Wide Spectrum HPV probe.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Pathology findings</em></p>\r\n<p>\r\n	The histological appearances of both tissues were typical of laryngeal papillomatosis. Microscopically, the papillo-mata appeared as projections of kerati-nised stratified squamous epithelium overlying a fibrovascular core (Fig.1a). Parakeratosis and koilocytes were present and the latter appeared as vacuolated cells with clear cytoplasm. There was no atypia seen in these tissues.</p>\r\n<p>\r\n	<em>In situ hybridisation</em></p>\r\n<p>\r\n	In situ hybridisation study detected specific signals of HPV-6 DNA in both cases and HPV-11 DNA in only the first case. The other viral types were not identified. The specific signal was observed as dense and homogenous, blackish purple nuclear stain, located in the superficial squamous epithelial cells (Fig. 1b&amp;c).&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<em>p53 and c-erbB-2 protein expressions</em></p>\r\n<p>\r\n	For the first case, tissue blocks from biopsy taken at onset and the latest biopsy in 2001 were selected after examination of the haematoxylin and eosin stained tissue sections. By immunohistochemistry, the first section showed positive staining for p53 protein, most pronounced in the lower third of the epithelium with focal inter-mediate staining, while section from the latest biopsy showed the staining had extended to the intermediate layer in a diffuse pattern (Fig. 2 &amp; 3).&nbsp;</p>\r\n<p>\r\n	For the second case, tissue blocks available from the biopsy taken at presentation showed patchy staining of p53 protein in the basal layer.</p>\r\n<p>\r\n	For both cases, immunohistochemical staining for c-erbB-2 were negative.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Juvenile laryngeal papillomas (LP) present in children or adolescents with multiple papillary tumours on the true cords, from which they spread to the false cords, epiglottis and subglottic area, and in rare cases even to the trachea and bronchi (Moore, 1959). The long period of papilloma infection of the larynx is reflected by the fact that our first patient required a tracheostomy at some stage of the disease. The more aggressive behaviour of the papilloma has also resulted in this patient requiring laser treatment.</p>\r\n<p>\r\n	Two types of HPV have been specifically associated with laryngeal papillomas. These are HPV-11, which was originally detected and cloned from a benign laryngeal papilloma and HPV-6 where it was isolated from a condyloma acuminatum (genital wart) and sub-sequently shown to be associated with condyloma acuminata and laryngeal papillomas (LPs) in adults and children (Gissman&nbsp; &amp;&nbsp;&nbsp; zur Hansen, 1980; Mounts et al, 1982). Both our cases were associated with HPV-6 and the first case had an additional HPV-11 which has been proved to be the most refractory to treatment (Hartley et al, 1994).</p>\r\n<p>\r\n	A variety of human tumours exhibit mutations in the p53 gene that can lead to increased stability of its protein, resulting in increased immunostaining (Lane, 1994). Overexpression of p53 protein is commonly demonstrable in benign epithelial lesions of the upper respiratory tract. A study showed that in LPs, overexpression of p53 protein increased with the grade of epithelial abnormalities. The distribution of positive cells changed from scattered and focal, in simple and abnormal hyperplasia, to diffuse in atypical hyperplasia (Luzar et al, 1997).</p>\r\n<p>\r\n	The HPVs produce two separate viral proteins, one of them an E6 viral protein, is not interactive with HPV-6 and HPV-11, and both are most frequently found in LPs (Knudson, 1993). Thus, in the presence of HPV-6 and HPV-11, p53 protein can still preserve its tumor-suppressor activity in LP and this may account for the significant lower rate of malignant transformation of LP (Luzar et al, 1997).</p>\r\n<p>\r\n	Another study identified 12 patients with recurrent laryngeal papillomatosis for a minimum of five years and each patient had at least three biopsies done; one biopsy was at the time of diagnosis, one at an intermediate time during treatment and the third from the last procedure (Rabah et al, 2000). They concluded that HPV typing,</p>\r\n<p>\r\n	especially HPV-11 may be helpful in identifying patients with aggressive recurrent laryngeal papillomatosis and the weak association between p53 indexes and procedures per year and virus type may have some predictive value in identifying the aggressive type. Our first patient had HPV-11 and was followed-up for nearly 15 years and histologically, so far, no atypical changes were seen in the epithelium.</p>\r\n<p>\r\n	Luzar et al showed c-erbB-2 protein was observed in the hyperplastic epithelium covering LPs, and staining increased with increasing grades of epithelial thickness (Luzar et al, 1997). Membranous staining is considered to indicate c-erbB-2 over-expression, which is characteristically associated with c-erbB-2 amplification in breast carcinomas. With the increasing grade of epithelial abnormalities, cyto-plasmic staining became predominant compared to membranous staining, and c-erbB-2 positivity sometimes occupied the whole thickness. Both our cases showed no membranous staining.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	One of the above cases followed-up had frequent recurrences and was shown to have HPV type 11 and p53 positive cells. These p53 positive cells were shown in the earlier specimen to be scattered and focally distributed, and follow-up biopsy after many years showed nearly diffuse distribution. These findings were similar as in other studies, where follow-up of the cases was recommended since they tend to recur (Luzar et al, 1997; Rabah et al, 2000).</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors would like to thank Encik Rosli Nasir of Hospital Universiti Kebangsaan Malaysia for preparing the photographs.</p>\r\n<p>\r\n	The study was supported by the Ministry of Science, Technology and environment (Grant IRPA No.03-037-03-075) and Universiti Kebangsaan Malaysia (F 4/99 &amp; FF/15/2001)</p>\r\n', 1),
(379, 376, '<p>\r\n	<strong>Introduction</strong></p>\r\n<p>\r\n	Renal cell carcinoma is a relatively rare tumour accounting for less than three percent of all adult cancers (Bennington JL and Beckwiht JB. 1975, Javadpour N. 1984). It is generally a tumour of adults where the average age at diagnosis is</p>\r\n<br clear="all" />\r\n<p>\r\n	between 55 to 60 years (Bennington JL and Beckwiht JB. 1975, Javadpour N. 1984). The clinical diagnosis of renal cell carcinoma is difficult since its clinical manifestations are quite variable; about 30 percent of patients have distant metastasis when the tumor is diagnosed (Bennington JL and Beckwiht JB. 1975, Javadpour N. 1984). The most common site of metastasis include lung, liver, bone, lymph nodes, adrenal and brain (Bennington JL and Beckwiht JB. 1975). These metastases may readily become targets for fine needle aspiration.</p>\r\n<p>\r\n	In this case report we present a 66-year-old gentleman with advance renal cell carcinoma in whom the diagnosis was made on fine needle aspiration cytology of para-aortic nodes containing metastatic deposits of renal cell carcinoma, and further supported by tissue biopsy of a cervical lymph node.</p>\r\n<p>\r\n	<strong>Case history</strong></p>\r\n<p>\r\n	A 66-year-old Chinese gentleman who was a known case of non insulin dependent diabetes mellitus for 14 years and hypertension, complained of intermittent left lumbar pain radiating to the left iliac fossa for two years. He was diagnosed to have lumbar spondylosis and treated with analgesics. He also complained of loss of appetite for two weeks before coming to the hospital but there was no weight loss. He had shortness of breath after climbing up four flights of stairs but there was no orthopnoea, paroxysmal nocturnal dyspnoea or chest pain. He had nocturia every two hours but no haematuria, poor flow or hesitation. There was no bowel symptoms, night sweats and fever. He was a chronic heavy smoker for about 40 years and smoked thirty cigarettes per day. On physical examination, the patient showed signs of heart failure with ankle oedema and basal end inspiratory crepitations. Abdominal examination revealed a&nbsp; non tender mass which was ballotable on the left lumbar region with ascites. Urine examination showed protein 4+, glucose 4+ and red blood cells 3+. The diagnosis of diabetic nephropathy with left renal tumour was made based on the above findings. Abdominal X-ray showed micro-calcifications at the lower pole of the left kidney which was consistent with scarring from a previous infection. Computed Tomography Scan showed multiple masses in the para-aortic region extending into the left renal hilum involving the upper pole of the left kidney. The radiologic&nbsp; impression was left renal lymphoma.</p>\r\n<p>\r\n	A left cervical lymph node measuring 2 x 1 cm which was firm and mobile was noted. Fine needle aspiration cytology of the para-aortic nodes was performed under ultrasound guidance using a 22-gauge, 20-cm-long-spinal-type needle which was connected to a 20-ml plastic syringe placed in a syringe holder.</p>\r\n<p>\r\n	Cytologic examination showed a cellular aspirate comprising of cohesive clusters of malignant cells with some attached to fibrin strands. The cells exhibit abundant cytoplasm with fine vacuolations (Figure 1). Occasional cells showed granular cytoplasm (Figure 2). The nuclei were moderately pleomorphic with some showing prominent nucleoli. Abnormal mitotic figures were also noted.</p>\r\n<p>\r\n	Excision biopsy of the left cervical lymph node showed infiltration by malignant cells forming multiple lobules in cohesive sheets separated by thin fibrous septae (Figure 3). Focal areas showing tubulopapillary arrangements were present. The cells displayed abundant pink to clear cytoplasm with distinct cytoplasmic border. The nuclei were large round and vesicular with prominent nucleoli and several mitotic figures were seen. Immunohistochemical study showed tumour cells were positive for cytokeratin, epithelial membrane antigen and vimentin. Marker for leucocyte common antigen was negative. A diagnosis of metastatic renal cell carcinoma of clear cell type was made.</p>\r\n<p>\r\n	X-ray of the lung showed nodules at the left lower zone adjacent to the lower rib, most likely a metastatic deposit. Ultrasound of the liver showed multiple nodules</p>\r\n<p>\r\n	consistent with metastases. There was no plan for surgery for him as the tumour was inoperable. The patient was referred to oncology unit for further management. He was admitted to the ward several times with the problem of widespread metastases including to the gut and lungs. He also presented with lower gastrointestinal bleeding and difficulty in breathing. He succumbed to his illness three years after the diagnosis was made.</p>\r\n<p>\r\n	<strong>Discussion</strong></p>\r\n<p>\r\n	Renal cell carcinoma is a rare tumour occurring more commonly in adults. Its usual presentations are haematuria (59%), flank pain (41%) and abdominal mass (45%). However, the combination of these three features, classically regarded as the diagnostic triad of renal cell carcinoma, occur in only nine percent of the patients and are usually a late manifestation (Skinner DG et al 1971). As many as 25 percent of renal cell carcinoma is asymptomatic, with discovery of the tumour being incidental to a routine physical examination and unrelated radiological study (Peterson RO 1986).</p>\r\n<p>\r\n	With the recent technologic advances in diagnostic radiology, percutaneous aspiration of renal lesions can be guided by several radiologic techniques include selective arteriography (Boijsen E and Link DP 1977), ultrasound (Holm HH et al 1975, Kristensen JK et al 1974),and CT scan techniques (Sundram M et al 1982) in an attempt to obtain diagnostic cytologic material. This radiologically guided aspiration was first introduced by Lindblom (Karp W and Ekelund L 1979) in 1952 and since then, fine needle aspiration biopsy for cytologic evaluation of renal masses has been performed more or less routinely. This minimally invasive diagnostic procedure is safe, economical and able to give fast diagnosis obviating open biopsy for tissue diagnosis.</p>\r\n<p>\r\n	Several studies had proven that fine needle aspiration cytology of a renal mass is a useful diagnostic procedure. With reference to malignancies, the usefulness of the method is related to its high sensitivity, 0.93, and high degree of typing accuracy(Murphy WM et al 1984, Pilotti S et al 1988). Reported success rates are 87 to 100 percent (mean of 93.5 percent) for renal tumours(Murphy WM et al 1984, Sundram M et al 1982).</p>\r\n<p>\r\n	The cytologic diagnosis of renal cell carcinoma by fine needle aspiration cytology poses no diagnostic difficulties whether the aspirates are from the primary site or metastatic deposits (Linsk JA and Franzen S 1984).This is because renal cell carcinoma displays fairly characteristic cellular features permitting their correct cytologic identification (Linsk JA and Franzen S 1984).</p>\r\n<p>\r\n	Aspirates from renal cell carcinoma are usually cellular even though in some cases the difficulty arises because of inadequate sampling or the presence of much necrotic debris and haemorrhage within the aspirates, obscuring the cellular details. This condition is particularly true for papillary variant of renal cell carcinoma because of the perculiar features of this variant that show presence of cystic and extensive degenerative changes (Pilotti S et al 1988).</p>\r\n<p>\r\n	Tumour cells may be single, or are arranged loosely in flat sheets, clusters, papillary fronds&nbsp; or an alveolar pattern. The smear pattern may occasionally be mixed. Three distinct cell types have been described on fine needle aspiration cytology (Koss LG et al 1984) , the clear cell, the granular cell and the oncocytic cell types, occurring either excusively or admixed together. The clear cells of renal cell carcinoma have abundant, fragile, finely vacuolated cytoplasm, best appreciated on the Diff-Quik stain. The cytoplasm is highly characteristic (Linsk JA and Franzen S 1984), as seen in this case particularly with Romanovsky stain and is termed &lsquo;opaque with or without vacuolization and granulation&rsquo; to distinguish it from the so-called clear cell of histopathology, which has been denuded of its cytoplasmic content by the histologic process. Cytoplasmic vacuoles are punched out, bubbly or lacy. The partially extruded nucleus is a useful identifying feature (Linsk JA and Franzen S 1984).The cell borders may be well defined but are usually indistinct. Granular cells have eosinophilic or cyanophilic cytoplasm, which is moderately dense and granular. Oncocytic cells have extremely dense, eosinophilic, compact cytoplasm with well-defined cell borders. Rarely, renal cell carcinoma may show a diffuse spindled appearance.</p>\r\n<p>\r\n	A nuclear grading system based on four nuclear grades defined in order of increasing nuclear size, irregularity and nucleolar prominence, proposed by Furhman et al (1982)&nbsp;showed that nuclear grade was effective in predicting survival and development of metastasis after nephrectomy (Amtrup F et al 1974). This grading system has been popular in routine surgical material of renal cell carcinoma and could readily be applied to fine needle aspiration cytology material. Several studies using Furhman&rsquo;s nuclear grading system showed high concordance between nuclear grading in fine needle aspiration material and histologic specimens (80 &ndash; 92% ) (Nurmi M et al 1984).These findings were further supported by the high interobserver and intraobserver reproducibility. Multiple grades coexist in about 15 percent of all renal cell carcinomas.&nbsp;</p>\r\n<p>\r\n	Even though most renal cell carcinomas are identified without much difficulties from fine needle aspirates; there are few diagnostic pitfalls that one may encounter especially when the aspirates are from metastatic sites. Renal cell carcinoma may metastasize to any site in the body including salivary gland, thyroid, ovary and soft tissue (Bennington JL and Beckwiht JB 1975). Therefore, cells of metastatic carcinoma should be distinguished from those of other tumours containing clear or granular epithelial cells that may arise in these organs (Bennington JL and Beckwiht JB 1975, Lever WF and Schaumberg-Lever G 1983, Nguyen GK 1986). The differential diagnosis of tumours with vacuolated (clear) cells includes acinic cell carcinoma and mucoepidermoid carcinoma of the salivary gland, clear cell thyroid carcinoma, clear cell tumour of the lung and mesonephric (clear-cell) carcinoma of the ovary. However, cells of renal cell carcinomas contain intracytoplasmic fat; therefore&nbsp; an oil-red-O staining of an air-dried aspiration smear is very helpful in their identification.</p>\r\n<p>\r\n	Metastatic renal cell carcinoma in the adrenal gland may pose a special problem since histologic differential diagnosis between renal cell carcinoma invading or metastasizing to the adrenal gland and a primary adrenal cortical carcinoma is very difficult if not impossible on the light microscopic level (Bennington JL and Beckwiht JB 1975). A similar degree of difficulty is to be expected in aspiration cytology of tumour cells aspirated from a renal cell carcinoma and those of adrenal cell carcinoma. However, adrenal cortical carcinoma tend to show more cellular anaplasia than do renal cell carcinomas (Bennington JL and Beckwiht JB 1975, Weiss LM 1984).Electron microscopic study of tumour tissue may be helpful in making a correct diagnosis.</p>\r\n<p>\r\n	In our case, the diagnosis was made on the aspirates taken from the para-aortic lymph node which was supported further with the biopsy of the cervical lymph node. Radiological imaging showed a right renal mass that gave a clue to the origin of the tumour.</p>\r\n<p>\r\n	In short, the presence of malignant epithelial cells arranged in cohesive clusters or sheets displaying vacuolated and granular cytoplasm in a patient with a previous history of radical nephrectomy for renal cell carcinoma is highly suggestive of metastatic renal cell carcinoma. The presence of a solid renal mass demonstrated by radiological imaging techniques and/or a vascular renal tumour demonstrated by angiography would also lend support to the diagnosis of a metastatic renal cell carcinoma.</p>\r\n', 1),
(380, 377, '<div>\r\n	<p>\r\n		<strong>Introduction</strong></p>\r\n</div>\r\n<div>\r\n	<p>\r\n		Myelofibrosis is a clonal myeloproliferative disease characterised by proliferation of predominantly granulocytic and megakaryocytic series in the bone marrow, associated with marrow fibrosis and extramedullary hemopoiesis. Peripheral blood dacrocytosis and leukoerythroblastic features are characteristic findings of myelofibrosis especially at the fibrotic stage. The pathogenesis of tear-drop poikilocytes and leukoerythroblastosis have been attributed to a variety of mechanisms from marrow fibrosis, extramedullary dyserythropoiesis or to the physical difficulties of red cells in traversing the tiny sinus wall apertures in the spleen.</p>\r\n	<p>\r\n		In 1977, DiBella et al had suggested that the spleen plays an important role in tear-drop poikilocytes formation after comparing the number of tear drop poikilocytes in patients with myelofibrosis before and after splenectomy. However, no subsequent studies were performed to look into the mechanisms involved in the formation of tear-drop poikilocytes and leuko-erythroblastosis. Manoharan et al (1998) described the effect of chemotherapy on the reduction of the extramedullary haemopoiesis and liver and spleen size as well as the reduction of tear-drop poikilocytes. Indirectly, this finding supported the theory that the spleen plays an important role in the formation of tear-drop poikilocytes. In this case report, we would like to highlight these peculiar findings that give insight to the possible mechanisms and the role of the spleen in the formation of tear-drop poikilocytosis and leukoerythroblastosis in myelofibrosis.</p>\r\n	<p>\r\n		<strong>Case Report</strong></p>\r\n	<p>\r\n		A 56 year-old lady was diagnosed to have primary myelofibrosis in 1999 in a private hospital. She was on regular follow-up with that hospital until the year 2002 when she was referred to Hospital UKM for the management of her increasing requirement of blood transfusion and massive splenomegaly.</p>\r\n	<p>\r\n		On physical examination, she was found to be afebrile. Her vital signs were normal. Examination of the cardiovascular system showed that she was in congestive heart failure with hepatosplenomegaly (liver 10cm, spleen 18cm below the costal margins). Laboratory investigations showed the following results: haemoglobin 6.7g/dl, WBC 24.6x10<sup>9</sup>/l, platelet 153x10<sup>9</sup>/l. Liver and renal profiles were normal, serum lactate dehydrogenase was 2425 U/l, serum uric acid 648mmol/l. In view of the worsening of the disease, she was started on low dose Busulphan (1mg daily) and was on regular follow-up. In July 2002, she developed two episodes of acute cholecystitis. Investigations done showed that her hemoglobin was 6.4g/dl, white cell</p>\r\n	<div>\r\n		<p>\r\n			count of 23.4x10<sup>9</sup>/l, platelet count 163x10<sup>9</sup>/l with leukoerythroblastic picture and tear-drop poikilocytosis (Fig 1A). Leukocyte alkaline phosphatase score (NAP) was 184/100 neutrophils. Ultrasound of the abdomen showed massive splenomegaly with multiple gall stones. In view of the frequent transfusion requirement and symptomatic massive splenomegaly, splenectomy and cholecystectomy was recommended. Following the operation in November 2002, she recovered without much complication. Review of her blood picture post splenectomy showed disappearence of the tear-drop poikilo-cytosis and post-splenectomy changes (Fig 1B). On the subsequent follow-ups, patient&rsquo;s peripheral blood films showed persistent reduction of the tear-drop poikilocytes with appearance of target cells, schistocytes, spherocytes and persistence of nucleated red cells.</p>\r\n	</div>\r\n	<p>\r\n		<strong>Discussion</strong></p>\r\n	<p>\r\n		Various mechanisms were postulated to be involved in the formation of tear-drop poikilocytes in myelofibrosis. One of it is the ineffective erythropoiesis in the bone marrow or extramedullary sites such as the liver and the spleen, and the other postulated mechanism is that these red cells become distorted or fragmented in their passage through the myelofibrotic marrow or the abnormal splenic channels. Leblond and Weed (1975) with their scanning electron micrograph observations in patients with myelofibrosis had concluded that the spleen plays a major role in the tear-drop poikilocytes formation. Following that, DiBella et al (1977) carried out a small study on the effect of splenectomy on tear-drop poikilocytosis in 13 patients with myelofibrosis; they found that the average number of tear-drop poikilocytes per 1,000 RBCs in the pre-splenectomy smears was 42 (range, 15 to 112), compared with 11 tear-drop poikilocytes in the post-splenectomy smears (range, 6 to 20 )(P = less than .001). Based on this fourfold decrease in the number of tear-drop poikilocytes post-splenectomy, they had again concluded that the spleen plays an important role in the formation of tear-drop poikilocytes. The postulated mechanisms in which the spleen may play a role in the formation of these tear-drop poikilocytes are: (i) the distortion of erythrocytes in their passage through the splenic sinusoids; (ii) the production of distorted erythrocytes by extramedullary sites in the spleen or a combination of both. Futher review of the literatures did not reveal any other reports on the study of mechanisms of how the spleen causes tear-drop poikilocytosis.&nbsp;&nbsp;</p>\r\n	<p>\r\n		Manoharan et al(1988) studied the effect of chemotherapy on tear-drop poikilocytes and other peripheral blood findings in myelofibrosis; the observed improvement of haemoglobin levels and red cell morphology with decreasing spleen size and splenic fibrosis after a course of chemotherapy. This study further supports the notion that spleen plays an important role in the red cell changes in myelofibrosis. However, the pathogenesis of the formation of tear-drop poikilocytes remains unclear. It would be interesting to look into the exact mechanism of the formation of tear-drop poikilocytes as well as leukoerythroblastosis in myelofibrosis.&nbsp;&nbsp;</p>\r\n	<p>\r\n		In this case, we have highlighted the interesting findings of reduction of tear-drop poikilocytes post-splenectomy and reinforced the important role of the spleen in the formation of tear-drop poikilocytes in myelofibrosis.</p>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(381, 378, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n	<p>\r\n		Adenocarcinomas account for 10&ndash;20% of invasive cervical carcinomas (Young RH et al 1990). Young and Scully (1989) first described a new subset of cervical adenocarcinoma that was designated as &lsquo;&lsquo;villoglandular papillary adenocarcinoma (VGPA) of the uterine cervix&rsquo;&rsquo; in 1989. The histological features of VGPA are exophytic growth, papillary architecture and mild to moderate cellular atypia. It has been reported as a distinctive histological subtype which developed in relatively young women (33-52 years old) and has a favourable prognosis (Young RH et al 1989, Hopson L et al 1990, Jones MW et al 1993, Reed W et al 1993, Costa MJ et al 1995). Approximately 80 cases of VGPA have been reported in the literature (Young RH et al 1989, Jones MW et al 1993, Kaku T et al 1997, Stanley-Christian H et al 1997, Khunamrnpong S et al 2001). Its incidence has been quoted as 3.7-4.8% of the adenocarcinoma of the cervix (Garcea A et al 2003). There are only six cases of VGPA with lymph node metastasis reported in the literature, of which five showed lymphatic space permeation in the main tumour (Kaku T et al 1997, Utsugi K et al 2002, Khunamornpong S et al 2001, Garcea A et al 2003). We report a case of VGPA with right obturator lymph node metastasis and a review of the six similar cases reported in literature.</p>\r\n	<p>\r\n		<strong>CASE REPORT</strong></p>\r\n	<p>\r\n		A 45-year-old multigravid Malay housewife presented with a one week history of generalised abdominal pain, which was intermittent, sharp stabbing in nature and aggravated by inspiration. She had two full term spontaneous vaginal deliveries that were antenatally uneventful. She was a known diabetic and hypertensive on metformin and prazosin for the past eight years. She never had a pap smear done. There was no family history of gynecological malignancy, smoking or alcohol consumption. There was no history of oral contraceptive usage.</p>\r\n	<p>\r\n		She was pale, and weighed 67 kilograms. Systemic examinations were unremarkable. Cervical examination revealed a nodular exophytic, friable, hemorrhagic growth, arising from the posterior wall of the cervix, measuring 2.0 cm in diameter. A piece of the tissue detached during manipulation. A biopsy was obtained, with the clinical impression of cervical carcinoma stage IB.</p>\r\n	<p>\r\n		Microscopically, the cervical biopsy specimen was composed of fragments of polypoidal tissues with villous and papillary fronds arranged in complex branching pattern. The papillae were lined by glandular epithelium displaying mild to moderate nuclear atypia (Figure 1). Mitotic figure count was brisk (90/10 high power field). Lymphovascular invasion was not present.</p>\r\n	<p>\r\n		She was planned for a Wertheim hysterectomy, however, during the operation, frozen section detected right obturator lymph node metastasis (Figure 2). Wertheim hysterectomy was abandoned due to the advanced disease, instead, a bilateral salphingoophrectomy, myomectomy for leiomyoma and right iliac and obturator lymphadenectomy were performed. She also received 30 doses of external radiotherapy with concurrent chemotherapy of five cycles of weekly cisplatin. Unfortunately she died seven months after the surgery due to chest infection.</p>\r\n	<p>\r\n		<em>A synopsis of the clinicopathological features of villoglandular papillary adeno-carcinoma of uterine cervix with lymph node metastasis in the literature</em></p>\r\n	<p>\r\n		There are approximately 80 cases of VGPA ever reported in the literature. Of these, six cases presented with lymph node metastasis (7.5%). A synopsis of the clinicopathological features of the above six cases together with our present case is listed in table 1. The ages ranged from 29</p>\r\n	<div>\r\n		<p>\r\n			to 54 years (mean, 41 years). The clinical presentation includes abnormal uterine bleeding (43%), vaginal discharge (14%), postcoital bleeding (14%) and abdominal pain (14%). Five cases had lymphovascular invasion (71%). The obturator lymph node (100%) is the commonest site of metastasis, with greater propensity towards the right side, followed by the external iliac lymph node (40%). The mitotic figure count reported by Utsugi K et al (2002) and the present case were 105/10HPF and 90/10HPF, respectively. All cases were followed up between seven months to 17 years, of which two died (28.6%), one due to the disease and the other because of chest infection.</p>\r\n		<p>\r\n			<strong>DISCUSSION</strong></p>\r\n		<p>\r\n			Villoglandular papillary adenocarcinoma is a distinct entity and rare histological variant of cervical adenocarcinoma, which is believed to have a favourable prognosis and predominantly develops in young women with oral contraceptive use (Young RH et al 1989, Jones MW et al 1993). Young and Scully (1989) described a case of oral contraceptive use in their 13 cases of VGPA. Later, Jones et al (1993) demonstrated a statistically significant difference in the incidence of oral contraceptive use in five of the 24 VGPA. Other published series showed no consistent association between oral contraceptive use and villoglandular papillary adenocarcinoma, hence it remains debatable (Garcea A et al 1988).</p>\r\n		<p>\r\n			The three important histopathological features of VGPA are 1) an exophytic growth pattern, 2) papillary architecture and 3) mild to moderate nuclear atypia. The epithelium can be a mixture of endometrial, endocervical and intestinal differentiation. The differential diagnosis of VGPA includes 1) serous papillary adenocarcinoma in which complex papillae lined by markedly atypical nuclei with high mitotic activity are present, 2) endocervical adenocarcinoma with a minor villoglandular component, and 3) minimal deviation adenocarcinoma, with a diffusely infiltrative growth pattern and absence of cytologic atypia.</p>\r\n		<p>\r\n			Young and Scully (1989) mentioned that VGPA should be reserved for those tumours composed exclusively or almost exclusively of villoglandular papillary pattern (Young RH et al 1989). Various authors have reported the association of other patterns, for example squamous cell carcinoma (Kaku T et al 1997), small cell carcinoma (Van Nagell JR et al 1988) and adenocarcinoma in situ (Jones MW et al 1993). Hence, a thorough search for coexistence of a different malignancy such as squamous cell carcinoma, small cell carcinoma, etc. is mandatory.</p>\r\n		<p>\r\n			In most malignancies, the tumour size, depth of invasion, lymphovascular invasion and lymph node metastasis are considered histological risk factors for recurrence. Utsugi K et al (2002) tried to determine the relationship between mitotic figure counts with incidence of lymph node metastasis. They found a significant difference in mitotic figure count of 105/10HPF in their patient with lymph node metastasis, as compared with 12 other patients without lymph node metastasis where the median mitotic figure count was 28/10 HPF (range between 4-70/10 HPF) (Utsugi K et al 2002).The mitotic figure count for our patient is 90/10 HPF, which supports this observation.</p>\r\n		<p>\r\n			Various reports recommend conservative management, i.e. a cone biopsy, after a careful review of the histopathological findings (Young RH et al 1989, Jones MW et al 1993). We emphasise that a careful search for any lymphovascular invasion, coexistence of a different tumour such as squamous cell carcinoma or small cell carcinoma and mitotic figure count be carried out, and when present, they should be included in the histopathological report. These should be considered as poor prognostic factors for VGPA.</p>\r\n		<p>\r\n			All the seven VGPA cases with lymph node metastases received radiotherapy and after a range of follow up of seven months to 17 years, six of the seven cases (86%) showed no evidence of disease. Although the number of cases is too small to give any conclusive comments, VGPA with lymph node metastases seems to have a better outcome after radical hysterectomy followed by radiotherapy and chemotherapy.</p>\r\n	</div>\r\n</div>\r\n', 1),
(382, 379, '<p>\r\n	Dear Editor,<br />\r\n	<br />\r\n	Tuberculosis (TB) is a disease which is becoming more common. It has various forms of presentation and a high index of suspicion is needed in all cases of uveitis.&nbsp;</p>\r\n<p>\r\n	<strong>Case Reports</strong></p>\r\n<p>\r\n	<em>Case 1</em></p>\r\n<p>\r\n	A 26-year-old Malay woman presented with a one-week history of painless decrease in right visual acuity. The right visual acuity was 6/36, N48. There was supero-temporal phlebitis with three yellow elevated choroidal lesions adjacent to it and cystoid macular oedema (Figure 1) . There was associated optic disc hyperaemia and mild vitritis. Fundus fluorescein angiography (FFA) did not reveal any capillary non-perfusion. The left eye was normal.</p>\r\n<p>\r\n	Her chest X-ray was normal and urine and sputum samples were negative for acid-fast bacilli. Her Mantoux reading was 15 mm. She was initially treated with streptomycin, isoniazid, rifampicin and pyrizinamide. After two days, the choroiditis improved but vitritis increased. She was started on a course of oral prednisolone (1mg/kg/day). Following further seven days of treatment, the phlebitis, choroiditis and vitritis resolved. The oral prednisolone was tapered off over two months. After a month, her vision had improved to 6/9, N5. She completed six months of anti-tuberculous therapy. She had no reactivation during anti-tuberculous treatment or after stopping prednisolone.&nbsp;</p>\r\n<p>\r\n	<em>Case 2</em></p>\r\n<p>\r\n	A 27-year old Malay woman with recent contact with TB patient had a two-week history of superior visual field blurring. Her visual acuity was 6/6, N5 bilaterally with no relative afferent pupillary defect. There was bilateral swollen discs with right vasculitis and adjacent retinitis (Figure 2).&nbsp; FFA revealed more than five disc diameters of capillary fallout distal to the arteritis and panretinal photocoagulation (PRP) was subsequently performed.</p>\r\n<p>\r\n	A brain CT scan was normal and connective tissue screen was negative. The Mantoux reading was 16mm. She was started on oral isoniazid, rifampicin, pyrizinamide and pyridoxime to which she responded rapidly during the first two days with a decrease in the area of retinitis. However by day seven, her mild vitritis had become worse even though the retinitis and vasculitis continued to improve. The vitritis responded to a course of oral corticosteroids of 40 mg/day which was tapered off over two months. She completed two months of all the drugs with the exception of rifampicin and isoniazid which were discontinued after nine months. There was no reactivation during treatment and for three months after stopping all medications. All clinical signs resolved.</p>\r\n<p>\r\n	<strong>Discussion</strong></p>\r\n<p>\r\n	In patients with retinal vasculitis, ocular TB should be a differential diagnosis (Morimura Y et al 2002). In addition to vasculitis, ocular TB may involve other structures such as the choroid and optic nerve.It is not unusual for ocular TB to occur in isolation with no evidence of systemic TB (Sarvananthan N et al 1998). Diagnosis is usually made by high index of clinical suspicion and significant Mantoux reaction (Morimura Y et al 2002).Samples of retinal vasculitic lesions are usually inaccessible but vitreous samples may be sent for polymerase chain reaction (PCR) (Sarvananthan N et al 1998). Monotherapy resistance rate has been reported as 7.3% (Pablos-Mendez A et al 1998)&nbsp; but may be as high as 28.7% (Khan MY et al 2001)for isoniazid. Hence the isoniazid test is less reliable now. The current regime is multidrug therapy where the reported resistance rate is still low at 2.2% (Pablos-Mendez A et al 1998). Corticosteroids and other immunosuppressive agents such as azathioprine may be used in conjunction with anti-tuberculous therapy when inflammation is marked. The vitritis in these cases may represent an immune response to breakdown products of the acid-fast bacilli (Morimura Y et al 2002). These cases illustrate the importance of not starting steroid alone when an infective process is suspected but to provide concurrent antibiotic cover. PRP may be required when there is occlusive vasculitis because rubeosis frequently occurs when ischaemia co-exists with inflammation.</p>\r\n', 1);
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(383, 380, '<p>\r\n	<strong>Hepatitis B virus (HBV)</strong></p>\r\n<p>\r\n	In 1965, Blumberg and co-workers discovered a unique antigen among the sera of Australian aborigines (Blumberg et al., 1965). They referred to this as the &ldquo;Australian antigen&rdquo;. Subsequent electron microscopy study by Dane et al. (1970) on the sera of antigen-positive patients revealed vast numbers of spherical and filamentous particles of 22 nm in diameter and also larger particles of 42 nm in diameter with central nucleocapsids and outer enveloping coats. With the discovery of the 42 nm particles, the term &ldquo;Dane particles&rdquo; was used. It was shown later that the Dane particles were in fact complete viral particles and were the causative agent for the liver hepatitis. Subsequently, the Dane particles were changed to a name which we are familiar with today, the hepatitis B virus (HBV). The 22 nm particles turn out to be the excess non-infectious surface protein complexes of the virus without the nucleocapsid cores.</p>\r\n<p>\r\n	<strong>Molecular background of hepatitis B virus (HBV)</strong></p>\r\n<p>\r\n	Hepatitis B virus (HBV) is a small enveloped virus which is 42 nm in diameter. Its partially double-stranded DNA genome of about 3.2 kb is enclosed by an icosahedral structure consisting of 180 or 240 HBV core proteins (HBcAg) (Bowyer and Sim, 2000).&nbsp;&nbsp; Enveloping this structure is a coat of lipid bilayer membrane studded with the HBV viral surface proteins (HBsAg).</p>\r\n<p>\r\n	&nbsp;&nbsp; The genome of HBV is unique in that it has overlapping reading frames. Thus it can encode about 50 % more polypeptides than would be expected from the size of its genome (Ganem and Varmus, 1987). The compact genome contains four reading frames which are the precore/core reading frame, preS/ S reading frame, polymerase reading frame and the X reading frame (Figure 1) (Kidd-Ljunggren et al., 2002). The compactness of the HBV genome is such that no non-translational region exists in it and the regulatory sequences reside inside the reading frames themselves (Torre and Naoumov, 1998).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The precore/core reading frame overlaps with the polymerase and X reading frames. In this reading frame, two start codons are situated, corresponding to the precore and core domains of the reading frame. The translation from first start codon across both the domains will produce the HBeAg.</p>\r\n<p>\r\n	On the other hand, translation of the core domain from the second start codon will produce the HBcAg.</p>\r\n<p>\r\n	&nbsp;&nbsp; As for the preS/ S reading frame, which overlaps with the polymerase reading frame as well, three domains exist (namely the preS1, preS2 and S domains) with three separate start codons defining the borders of the respective domains. This arrangement permits the preS/ S reading frame to produce the HBsAg of three different sizes. The largest of these, called the L HBsAg, contains the preS1, preS2 and S domains (Heermann and Gerlich, 1991). Translation from the second start codon will produce a surface protein of intermediate size (referred as M HBsAg) which contains both the preS2 and the S domains. The smallest of the surface protein (referred as S HBsAg) consists of only the S domain and is translated from the third start codon.</p>\r\n<p>\r\n	&nbsp;&nbsp; The other two frames correspond to the viral polymerase and X proteins respectively, as indicated by the names of the reading frames. The polymerase reading frame is the largest of all the reading frames found in the HBV genome and it partially overlaps all the other reading frames (Kidd-Ljunggren et al., 2002). For the viral DNA polymerase, besides having the function to replicate the viral DNA, other catalytic domains are included such as the primase, reverse transcriptase and RNase domains (Idilman et al, 1998). All of these catalytic domains are involved in the vital role of replicating the viral genetic material. Briefly, viral replication is initiated with the transcription of the HBV genome to produce a 3.4 kb pregenomic RNA (pgRNA). This RNA material is longer than the genome itself due to the presence of a redundant short terminal (Kidd-Ljunggren et al., 2000).</p>\r\n<p>\r\n	&nbsp;&nbsp; After translation of the viral polymerase, the protein will automatically attach itself to the 5&rsquo;-end of the pgRNA. This event will initiate the packaging process of the viral pgRNA, together with the attached polymerase, into the nucleocapsid struc-ture (Wang et al., 1994). This process requires the presence of some host factors such as the molecular chaperone complex of hsp90 (Hu and Seeger, 1996). The viral DNA synthesis will occur inside the nulecocapsid once packaging is comple-ted. The primase domain of the viral polymerase has a role in initiating the DNA replication process through reverse transcription where the pgRNA is utilized as the template. During the synthesis of the minus-strand DNA, the RNase domain of the polymerase will degrade the whole pgRNA except for a small segment at the 5&rsquo;-end. The undigested 5&rsquo;-end RNA segment will function as the primer for the subsequent synthesis of the complement-tary plus-strand DNA. However, before the plus-strand DNA synthesis is completed, the viral particle will become mature through the acquisition of its outer enve-lope coat consisting of a lipoprotein membrane studded with the HBsAg of various sizes. This fact explains the uniqueness nature of the HBV genome, which is partially double-stranded. The viral polymerase will remain attached to the viral DNA genome, and so be incorporated into the viral nucleocapsid as well. Once infec-tions of the host cells occur, the released partially double-stranded HBV genome will resume its synthesis of its outstanding plusstrand DNA segment, resulting in a final complete double-stranded DNA genome.</p>\r\n<p>\r\n	&nbsp;&nbsp; Coming to the last polypeptide encoded by the HBV genome, HBx is a small protein (154 amino acids) which was reported to be involved in the modulation of host biological processes (Murakami, 1999; 2001). Some hosts factors that are involved in the interactions with the HBx are the transcription factors of p53 and Smad4 (Elmore et al., 1997; Lin et al., 1997; Lee et al., 2001). However, there is still much to learn about this protein, especially its role in the development of hepatocellular carcinoma in infected hosts.</p>\r\n<p>\r\n	<strong>Epidemiology and pathogenesis of HBV</strong></p>\r\n<p>\r\n	HBV is one of the most common viral pathogen affecting the world population. Approximately 350 million people around the world are infected with the virus (Purcell, 1993). It is estimated that 50 million new cases are observed annually with 5 to 10 % of them being adults while the remaining consists of children (Mayerat et al., 1999). Infections by HBV can result in acute and chronic hepatitis diseases. The figure of deaths attributed to HBV infection is estimated at 1 million people per year (Thomas and Jacyna, 1993). Although all body secretions and excretions have been found to contain HBsAg, only blood, semen, menstrual and vaginal fluids have been shown to be infectious (Hollinger and Liang, 2001).</p>\r\n<p>\r\n	&nbsp;&nbsp; When infected, the occurrence of liver hepatitis is not caused directly by the HBV per se but due to the host own immune response. When HBV infection occurred, the host immune response is stimulated where the presence of HBV-specific CD8+ cells can be detected in infected subjects. As HBV does not directly cause the destruction of the hepatocyte it invades, the infected hepatocyte acts as a reservoir for further infection of other similar host cells. This permits the HBV-infected hepatocytes to continuously shed the viral particles into the blood stream, thus ensuring that all the hepatocytes in a particular individual are infected (Seeger and Mason, 2000). Infected hepatocytes will display the immunodominant T cell epitopes presented by HLA class I mole-cules on their surfaces (Chisari and Ferrari, 1995). This acts as the targets for the CD8+ cells to the infected hepatocytes, thus resulting in the destruction of the infected hepatocytes.</p>\r\n<p>\r\n	&nbsp;&nbsp; Infections by HBV can lead to either acute or chronic hepatitis. Acute infection usually runs within a period of between 1 to 6 months, which includes the incubation period of between 8 to 20 weeks (Webster and Bertoletti, 2002). The clinical symp-toms that developed following acute infection are fever, severe malaise, loss of appetite and development of jaundice. Most individuals with acute hepatitis B will recover with clearance of viral antigens and HBV DNA in their sera (Yuen and Lai, 2001). This is possible when the host immune system is strong enough to overcome the viral infection. It is believed that resolution of the acute hepatitis B occurs when adequate productions of B and broad-based CD8+ T cells producing protective antibodies targeting the epitopes of the viral proteins are achieved (Bertoletti et al., 1991; Nayersina et al., 1993). Besides that, suppression of the viral infection is also performed by cytokines (especially a and g interferons) and tumor necrosis factor (TNF-a) which play a part in destruction of the viral replicative inter-mediates such as viral proteins, the replicating DNA and RNA (Tsui et al., 1995). Individuals who recovered from acute hepatitis B will be conferred with life-long immunity against the virus. However, for those who are suffering from acute hepatitis B, there is a 1 % chance that the conditions for these individuals will dete-riorate and develop into fulminant hepatitis (Lee, 1993). Fulminant hepatitis is cha-racterized by liver failure which is very often fatal. The failure of the liver functions is due to sudden massive immune-mediated lysis of the infected hepatocytes without giving enough time for the liver to recuperate (Gove and Hughes, 1991; Leifeld et al., 2002). A recent study speculated that vigorous replication of HBV might be a factor involved in the development of fulminant hepatitis (Inoue et al., 2006).</p>\r\n<p>\r\n	&nbsp;&nbsp; The other form of liver disease that HBV infection can lead to is chronic hepatitis B. Chronic hepatitis B is characterized by high HBV DNA levels, high risk of trans-missions, elevated liver enzymes such as alanine aminotransferase (ALT) in the sera and high risk of developing liver cirrhosis and liver cancer, hepatocellular carcinoma (HCC) (Lok and McMahon, 2001). The activity of viral replication in patients with chronic hepatitis B is relatively high where it is estimated that 1 x 1011 HBV particles are released into the blood circulation everyday (Nowak et al., 1996).</p>\r\n<p>\r\n	&nbsp;&nbsp; Chronic hepatitis B could occur when the rate of infected liver cells death is balanced by HBV infections of new liver cells due to the host immune response being not vigorous enough to clear the infection. The responses of cytotoxic T cells and HLA class H restricted T cells against the viral nucleocapsid antigens are weak in chronic patients (Chisari and Ferrari, 1995). One of the contributing factors of chronic hepatitis B is the long life-span of hepatocytes with an average half-life of 6 to 12 months. The stable hostvirus interactions also play a role in the persistence of chronic hepatitis B. The weak host immune response usually leads to scarring, disruption of blood flow and obstruction of bile drainage of the liver organ, thus interfering with the vital liver functions.</p>\r\n<p>\r\n	&nbsp;&nbsp; Given the major role plays by host immune system on the development of chronic hepatitis B, this liver disease is most often incurred by individuals with weak immune system. It has been estimated that about 90 % of newborns to mothers positive for HBeAg will develop chronic hepatitis B (Beasley et al., 1983). Besides that, about 25 to 30 % of infants and children under the age of 5 years old will develop chronic hepatitis in their life through contacts with persons infected with HBV, especially their mothers (Beasley et al., 1982; McMahon et al., 1985). New-borns and young infants are especially vulnerable because of the immaturity of their immune systems. Besides infants and children, immunosuppressed individuals such as those infected with HIV-1 or on maintenance haemodialysis have a greater risk of developing chronic hepatitis B than healthy persons when exposed to HBV (Bodsworth et al., 1989; Horvath and Raffanti, 1994).</p>\r\n<p>\r\n	&nbsp;&nbsp; There is a great chance that chronic hepatitis B patients will eventually die from liver injuries such as cirrhosis or HCC, which is estimated to be about 25 to 40 %. Out of these, the fatal rates are 50 % for male and 15 % for female (Lau et al., 1997). It has been estimated that chronic hepatitis B patients have a 200 to 300 fold of increased risk of developing HCC than the general population (Maynard, 1990).</p>\r\n<p>\r\n	&nbsp;&nbsp; In some individuals infected by HBV, no discernable symptoms for either acute or chronic hepatitis B can be observed. These individuals are grouped as the &ldquo;asymp-tomatic carriers&rdquo;. The sera from these individuals are positive for HBsAg but have normal or minimal raised liver enzymes such as ALT (Torbenson and Thomas, 2002). They can be diagnosed to be negative for HBeAg and positive for anti-HBe with undetectable or low HBV DNA level (Lok and McMahon, 2001). Long-term follow-up studies showed that asymptomatic carriers have lower risk of developing liver diseases like cirrhosis or HCC and they are less likely to infect other individuals (Lindh et al., 2000; Hsu et al., 2002; Martinot-Peignoux et al., 2002). However, about 20 to 30 % of them can reacquire hepatitis B disease where elevated biochemical indicators, high HBV DNA level and sero-reversion from anti-HBe back to HBeAg can be observed (de Franchis et al., 1993; Bellentani et al., 2002; Hsu et al., 2002). Repeated reactiva-tions of hepatitis B or a sustained one can result in liver injury but frequently symp-toms may be absent.</p>\r\n<p>\r\n	<strong>Prevalence of HBV infection in Malaysia</strong></p>\r\n<p>\r\n	HBV infections are endemic in Asia, South Pacific, sub-Saharan Africa, Middle East and the prevalence are particularly high in some indigenous populations such as the Aborigines and Native American Indians in South and Central Americas (Maynard, 1990; McMahon, 1997; Mast et al., 1999). It is estimated that 75 to 80 % of the HBV infection occur in the Western Pacific and in the South East Asia regions (Gust, 1996; Merican et al., 2000). The high endemicity in these regions is thought to be due to mother-to-child perinatal transmission (Simmonds, 2001).</p>\r\n<p>\r\n	&nbsp;&nbsp; In Malaysia, one of the earlier studies on HBV was performed by Ton et al. (1979). In the study, it was found that HBsAg and anti-HBc in blood donors from the Kuala Lumpur hospital was 5.5 % and 50.1 % respectively. When comparison of the HBsAg-positivities between the three major races in the Malaysian population (Malay, Chinese and Indians) was made, no significant difference was found between them.</p>\r\n<p>\r\n	&nbsp;&nbsp; A study on Malaysian intravenous drug users by Mangalam et al. (1986) observed that the rate of HBV infections among the drug abusers were similar to the general population. Thus, this could indicate that intravenous drug injections might not be a major route of transmission for HBV in Malaysia. As in the general case for the Asia-Pacific region, the main routes of HBV transmission among the Malaysian popu-lation could be due to mother-to-child peri-natal transmission as well.</p>\r\n<p>\r\n	&nbsp;&nbsp; In Malaysia, the prevalence of HBV infection incidence has been significantly reduced since the introduction of a nationwide vaccination program in 1989 (Ng et al., 2005). The program, called the Expanded Program of Immunization, invol-ved three doses of vaccination for the newborns. The first dose was given to a newborn soon after birth followed by the second dose 1 month later and 5 months later with the third dose. A particular per-son is considered to be protected against HBV when an anti-HBs titer of at least 10 mIU/ mL is detected in his or her serum (Ng et al., 2005). Prior to the implement-tation of the vaccination program, a study performed by Ton and Noriah (1987) on 653 healthy Malaysian individuals of both sexes revealed that 29.6 % of them were positive for the anti-HBs. This is comparable to a report on 3807 unvac-cinated Malaysian school children before the program was started where 27.4 % was found to be positive for the anti-HBs (Ng et al., 2005). After the implementation of the program, a statistical analysis on 97786 school children vaccinated from 1989 to 1996 showed that 59.3 % of them were positive for the anti-HBs and had humoral immunity against HBV (Ng et al., 2005). The successful result of this vaccination program saw a reduction from 2.7 % to 0.4 % of HBsAg carriers among school children during the study period.</p>\r\n<p>\r\n	&nbsp;&nbsp; However, despite the implementation of the vaccination program, a significant proportion of the Malaysian population is still afflicted with HBV. Merican et al. (2000) reported that about 75.3 % of the chronic hepatitis diseases suffered by patients is caused by HBV. A study on 17048 healthy Malaysian volunteers (mean age, 34 years) found that 893 were positive for HBsAg (Merican, 1999). Taking this study as a representative figure of the current HBV infection in the Malaysian population, it is estimated that about 5.24 % are struck down with chronic hepatitis B. When stratified according to the ethnic composition of the Malaysian population, the Chinese incurred the highest proportion of HBV infections, followed by the Malays and Indians (Yap, 1994; Merican et al., 2000). When compared with the study done by Ton et al. in 1979, it can be observed that the current level of the Ma-laysian population positive for HBsAg is comparable to that found in a sample population of more than 20 years ago. However, it seems that the demography of infection between the different races has changed since the study was performed in 1979.</p>\r\n<p>\r\n	&nbsp;&nbsp; The infections by HBV in Malaysia have created a major healthcare problem as acute and chronic hepatitis B patients require substantial medical attention. In a study to determine the causes of jaundiced cases surveyed among patients admitted into the hospitals in Kelantan and Terengganu in 1996, Saat et al. (1999) found that 1.4 % of the cases could be traced to infection by HBV. Due to their frequent close proximity with HBV carriers, health care workers are at an increased risk to exposure to HBV. A study among the health care workers in Negeri Sembilan in 1989 observed that a substantial number of the health care workers were positive for the serological markers for HBV (Tan et al., 1992). The study involved 2500 health-care workers of which about 25 % were found to be positive for the serological markers. Further stratification revealed that 2.1 % had HBsAg while 22.8 % were positive for anti-HBs. However, for the latter cases, it was not known whether the positivity for anti-HBs was due to history of HBV infection or to vaccination. Given this situation, it is imperative that steps should be taken to vaccinate the health-care workers in order to offer them protection against HBV. In lieu of this, the Universal Precautions Policy guidelines were adopted for health care workers. To find out if the policy was properly implemented, a survey on the protective status against HBV was performed on 625 subjects comprising health-care workers and under-graduate students undergoing clinical training in two hospitals in Kuala Lumpur in 2001 (Hesham et al., 2005). It was ob-served that only 58.4 % of the subjects had taken a complete HBV vaccination course while 82.2 % of the subjects had only been vaccinated with a single dose. HBV infections could also increase the burden faced by the current health-care system. HBV-contaminated blood could cause in-fections among patients seeking treatment for other types of illnesses. For example, in a report by Menon and Aiyar (1997), it was mentioned that about 20% of the 55 paediatric oncology patients who received blood products at Hospital Universiti Sains Malaysia in Kota Baru were positive for HBsAg. Besides that, the treatment costs for HBV infections are expensive. The current regime of anti-viral treatments administered for patients infected with HBV in Malaysia are the a-interferon, lamivudine and thymosin a-1 (Mohamed et al., 2004). Besides it was observed that about 80 % of the HCC cases observed in the country were caused by HBV (Yap, 1994).</p>\r\n<p>\r\n	<strong>HBeAg / anti-HBe productions and the associated mutations</strong></p>\r\n<p>\r\n	HBeAg is a viral protein that does not form a part of the structure of the viral particles but shares about 90 % of the amino acid sequence with the HBcAg. This is because the HBeAg is translated from the first start codon of the precore/ core reading frame, resulting in larger precursor protein of HBeAg with an extra N-terminal domain of 29 amino acid residues. The first 19 amino acid residues of the extra N-terminal functions as a signal peptide to guide the precursor HBeAg to the host endoplasmic reticulum compartment to be processed. Processing of this immature protein in the endoplasmic reticulum will result in the cleavage of the 19 residues from the precursor HBeAg. Subsequently, the C-terminal domain will be cleaved off as well at the arginine-rich region, thus resulting in a shortened mature form of HBeAg which is then secreted out from the host cells (Lau and Wright, 1993). Some of the mature HBeAg is localized on the host cell membrane while the rest is secreted into the blood stream. In comparison, the HBcAg does not have the N-terminal domain and retains its C-terminal domain. Despite the high similarity between these two viral proteins, their tertiary structural conformations are quite different. This is due to the presence of an intramolecular disulfide bond in the HBeAg (Nassal and Rieger, 1993; Wasenauer et al., 1993).</p>\r\n<p>\r\n	&nbsp;&nbsp; HBeAg is utilized as a serological marker for the infectivity of HBV, in particular individuals, in clinical diagnosis (Magnius and Espmark, 1972; Shikata et al., 1977). HBeAg is usually detected in the serum when HBsAg begins to appear. The presence of HBeAg in the serum correlates with the presence of viral activity in the liver. In the diagnosis of acute hepatitis, HBeAg does not play an important role as a diagnostic marker because the antigens are cleared within weeks after the acute disease has resolved. However, in chronic hepatitis B, this antigen is important as an indicative marker as HBeAg can remain detectable for a sustained period of time, usually in terms of months or years (Merican et al., 2000).</p>\r\n<p>\r\n	&nbsp;&nbsp; The function of HBeAg with respect to the virus is not fully understood yet. In an <em>in vitro </em>study, the absence of HBeAg does not seem to impede replicative ability of the virus (Tong et al., 1991). It is speculated that HBeAg might play a role in tricking the immune defense system of the infected host. It has been proposed that the expres-sion of HBeAg during perinatal infection can induce immune tolerance of the HBV by the host and that the HBeAg acts as a buffer to attack by anti-HBc on infected hepatocytes. This reasoning is based on the high sequence similarity between HBeAg and HBcAg (Tong et al., 2005).</p>\r\n<p>\r\n	&nbsp;&nbsp; Nevertheless, HBeAg positivity in HBV-infected patients has been found to be one of the risk factor for the development of liver cirrhosis (Liaw et al., 1988; Realdi et al., 1994; Yu et al., 1997). In cirrhotic patients, the presence of HBeAg is one of the risk factors in the occurrence of liver decomposition (Fattovich et al., 1995). It was reported that patients with decom-pensated cirrhosis who were negative for HBeAg have a better 5 year survival rate than those who were positive for the antigens (de Jongh et al., 1992). A reduce-tion in the risk of developing hepatic decompensation and survival improvement can be achieved after the clearance of HBeAg, either spontaneously or after undergoing anti-viral treatments (de Jongh et al., 1992).</p>\r\n<p>\r\n	&nbsp;&nbsp; With the disappearance of HBeAg from the serum, seroconversion to anti-HBe can occur. The presence of anti-HBe in the early phase of acute hepatitis is an indication of the resolution of the acute infection. On the other hand, in chronic hepatitis B, the disappearance of HBeAg and appearance of anti-HBe is associated with improved biochemical and histological standing of the liver disease.</p>\r\n<p>\r\n	&nbsp;&nbsp; Although anti-HBe positivity in HBV-infected patients signifies clinical im-provement, in some cases liver damage may still persist with active HBV DNA replication (Hadziyannis, 1995). Even after seroconversion to anti-HBe, reactivation of viral replication can still occur where seroreversion back to HBeAg positive status can happen.</p>\r\n<p>\r\n	&nbsp;&nbsp; Besides that, mutations in the HBV genome affecting HBeAg productions can also result in the emergence of HBeAg-negative mutants. Two of the most pro-minent mutations in these HBeAg-negative mutants are the A1762G1764&rarr; T1762A1764 core promoter mutations and the precore stop codon mutation. The A1762G1764&rarr;T1762A1764 dual muta-tions occur in the core promoter region that regulates the transcription of both the precore-mRNA, which is the template for HBeAg and the pgRNA (Figure 2) (Yuh et al., 1992; Takahashi et al., 1995). The precore stop codon mutant is characterized by a G to A nonsense nucleotide mutation at nucleotide 1896 of codon 28 of the HBeAg (TGG&rarr;TAG) which can result in the truncating of the translation of HBeAg into a short peptide of 28 amino acids (Figure 3) (Brunetto et al., 1989; Carman et al., 1989; Tong et al., 1990).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Besides these two mutations, the base variants at nucleotide 1858 in codon 15 can affect HBeAg production as well. The common variants exist at the aforemen-tioned site are the C1858/ T1858 variants. These variants are not directly involved in the truncation of the HBeAg production per se as conversion from one variant to the other will result in a silent mutation. However, they affect the HBeAg production through their pairing to nucleotide 1896 of the codon 28 in the secondary loop structure of the pgRNA (Figure 3). This loop is a hair-pin structure which functions as an encapsidation signal. This structure is essential for recruitment of the HBV polymerase which in turn is essential for the synthesis of the HBV genome and viral replication. Disruption of this structure is detrimental to the HBV replication. In the hair-pin secondary structure of the encapsidation signal, the nucleotide at 1858 is intimately paired to nucleotide 1896 through base pair bonding. Thus, the fate of precore stop codon mutation (TGG&rarr;TAG) is dependent upon the nucleotide at 1858. When cytosine is the nucleotide in place at nucleotide 1858 (hence referred as the C1858 variant), stable formation of the precore stop codon mutant will be prevented (Figure 3). This is due to the reason that incompatible pairing of adenine at 1896 with cytosine at nucleotide 1858 will confer a weak binding force to the encapsidation signal structure.</p>\r\n<div>\r\n	<p>\r\n		&nbsp;&nbsp; This will cause a disruption to the all important structure and so prohibiting the replication of the HBV mutant. On the other hand, if the nucleotide at 1858 is a thymine (hence referred as the T1858 variant), the formation of the precore stop codon mutant is conducive as the thymine can stably pairs with adenine at nucleotide 1896. Besides that, TGG wild-type can exist as well because the thymine at 1858 can wobble pair with guanine at nucleotide 1896 without disrupting the essential encapsidation signal structure (Lok et al., 1994).</p>\r\n	<p>\r\n		&nbsp;&nbsp; In Malaysia, an early study performed by Ton et al. (1996) on 43 HBsAg Malaysian carriers showed that infections by the C1858 variants were quite substantial. In the study, 4 of the 43 samples (9.3 %) were found to be of precore stop codon mutants. Of interest was that two of the mutants were found to co-exist with the TGG wild-type. In another study, Yap et al. (2002) detected 13 precore stop codon mutants out of 33 HBeAg-positive and 32 mutants out of 48 HBeAg-negative Malaysians sampled. In comparison to a study outside the country, Okamoto et al. (1994) observed a staggering high frequency of about 90 % of the mutants in patients positive to anti-HBe. Yap et al. (2002) had also performed sequencing analyses on the HBeAg-negative samples which were not infected by the mutants. The sequencing analyses revealed that 5 of them had the A1762G1764&rarr; T1762A1764 dual mutations while another sample had mutation at the start codon. On top of that, they found a significant association between T1858 variants with the precore stop codon mutations, which concurs with many other studies.</p>\r\n	<p>\r\n		&nbsp;&nbsp; In another study conducted in Malaysia, it was observed that among the sera samples from 44 HBV-infected patients, the prevalence of the core promoter mutations and precore stop codon muta-tions were determined to be 56.8 % and 9.1 % respectively (Ton et al., 2002). Intriguingly, in their study, it was observed that the number of patients with HBeAg-positive status was higher than those with anti-HBe-positive status when those in-fected with the core promoter mutants were taken into account. In comparison to a study on Brazilian HBV carriers with anti-HBe positive status, only about 24 % of them were found to be infected with the precore stop codon mutations (Castro et al., 2001). They reasoned that this was due to the large majority of the subjects being infected by the C1858 variants which, as discussed above, would preclude the stop codon mutation.</p>\r\n	<p>\r\n		&nbsp;&nbsp; However, in a later study on HBV-infected Malaysian patients, only low numbers of the above mutants were observed (Lim et al., 2006). In the study, only 11 out of 77 (14.3 %) HBV-infected patients examined were found to be infected with the core promoter mutants while 7 (9.1 %) were co-infected by both wild-type and the mutants. Interestingly, no significant correlation was observed between the core promoter mutations and the HBeAg/ anti-HBe status in this study. This contradicts with a few other studies in other part of the world where such correlations were observed (Hussain et al., 2003; Okamoto et al., 1994). Despite this anomaly, many other studies did not observe such correlations (Fran&ccedil;a et al., 2004; Lindh et al., 1999, Orito et al., 2001, Takahashi et al., 1995). One possible explanation for this is that the occurrences of T1762A1764 mutations only decrease the productive capability of HBeAg of the particular HBV strain but not totally abolishing it. It was shown by Parekh et al. (2003) that T1762A1764 mutations could only reduce the HBeAg production by about 20 %.</p>\r\n</div>\r\n<div>\r\n	<p>\r\n		&nbsp;&nbsp; Of interest is the observations of sera which were positive for both HBeAg and anti-HBe in two of the sera samples in the Malaysian study (Lim et al., 2006). This was also encountered in an earlier study by Ton et al. (1995) on sera taken from some</p>\r\n	<p>\r\n		Malaysians infected with HBV. The status of positivities for both HBeAg and anti-HBe might signify the ongoing process of sero-conversion whereby the HBeAg serological marker was not completely eliminated from the blood circulation yet.</p>\r\n	<p>\r\n		&nbsp;&nbsp; Also, in the study by Lim et al. (2006), no precore stop codon mutations were observed among the HBV samples examined. The study also showed that of the 46 samples that were successfully assayed, 33 of them were infected by the T1858 variants while 13 were infected by the C1858 variants. This was in concordance to an earlier study by Ton et al. (2001) where the occurrences of the T1858 variants significantly predominate over the C1858 variants (82.1 % vs. 14.8 %).</p>\r\n	<p>\r\n		&nbsp;&nbsp; Due to the lack of any precore stop codon mutant in the study by Lim et al. (2006), no correlation study between T1858/ C1858 variants of codon 15 with nucleotide 1896 of codon 28 was performed. However, co-infections of the precore stop codon mutants and the wild-type strains could still be observed among the Malaysian HBV carriers (Ton et al., 2001). The contrasting results observed between the various studies conducted in Malaysia could indicate that the distribution of the HBV mutants among the Malaysian population is not permanent but varies in time as well as in locations.</p>\r\n	<p>\r\n		&nbsp;&nbsp; Other than the core promoter mutations and the precore stop codon mutations, there are other less well-known mutations which could reduce the production rate of HBeAg. It was shown by Parekh et al. (2003) that, in combination with the T1762A1764 mutations, mutations at nucleotides 1753 and 1766 could result in a decrease in HBeAg production by up to 80 %. Besides that, some mutations occurring downstream of the precore start codon can decrease HBeAg translation too (Ahn et al., 2003). In addition to these, mutations such as at the precore start codon or TAA stop codon mutation at codon 2 could also abolish the production of HBeAg (Lindh et al., 1996). These mutations could explain for the presence of HBV in anti-HBe-positive patients where both the core promoter and the precore stop codon mutations were not detected.</p>\r\n	<p>\r\n		<strong>Association of mutations affecting HBeAg productions with liver disease prognosis</strong></p>\r\n	<p>\r\n		Although the mutations mentioned previously are associated specifically with HBeAg productions, many studies found that they could be related to the prognosis of liver diseases as well. The A1762G1764&rarr;T1762A1764 dual muta-tions were frequently observed in patients with chronic hepatitis but were rarely found in asymptomatic carriers (Okamoto et al., 1994; Kim et al., 1999). These mutations were also associated with increased inflammation and fibrosis in both HBeAg-positive and HBeAg-negative HBV infected chronic hepatitis patients (Lindh et al., 1999). The mutations had also been found to be associated with fulminant hepatitis in a few studies (Sato et al., 1995; Friedt et al., 1999). These mutations do not affect the transcription of HBV pgRNA from which the viral core peptides are translated (Okamoto et al., 1994). With the occur-rences of these mutations, the equilibrium balance between HBeAg in the blood circulation and the core peptides presented by the class 1 HLA molecules on the sur-face of infected hepatocytes will be eliminated. As HBeAg has been shown to have suppressive effect on T helper cells, its down-regulation may explain the asso-ciated clinical prognosis observed. The core peptide has been determined to contain most of the primary sequence of HBeAg. Thus, it is possible that circulating HBeAg may prevent cytotoxic T lympho-cytes from recognizing infected hepatocytes which display the core peptides on their surface by their HLA molecules. This is significant because cytotoxic T lymphocytes may be directed to the infected hepatocytes that display the core-derived peptides and so causing injury to them (Mondelli et al., 1982; Bertoletti et al., 1991). The decreased synthesis of HBeAg could free some of the cytotoxic T lymphocytes from their suppressive effect and so a greater number of the lymphocytes may be directed toward the infected hepatocytes thus resulting in greater liver injury (Takahashi et al., 1995). One study has shown that HBcAg is able to activate a more vigorous immune response than HBeAg <em>in vivo</em>, thus causing more severe liver damage (Nishizono et al., 1997).</p>\r\n	<p>\r\n		&nbsp;&nbsp; As mentioned, the disruption of HBeAg production could result in the withdrawal of hepatocyte display of HBeAg-derived cyto-toxic CD8+ T cell epitopes but viral replication will not be affected (Carman et al., 1993). In a study by Yuasa et al. (2000), it was found that most Vietnamese patients with fulminant hepatitis carried the precore stop mutation while none was found in patients with acute non-fulminant hepatitis. This is in concordance to an earlier study where similar observation was made (Omata et al., 1991). Transmission of these HBV mutants may lead to development of fulminant hepatitis in the newly-infected individuals (Yotsumoto et al., 1992; Liang et al., 1994). Besides, mutation in codon 28 could render the HBV strain with enhanced encapsidation advan-tage that makes them more infectious and causes hepatitis when they do so (Scaglioni et al., 1997).</p>\r\n	<p>\r\n		&nbsp;&nbsp; Lai et al. (1994) reported that mutations at codon 28 were associated with most anti-HBe seroconversion. Generally, the occurrences of mutations at the precore stop codon before seroconversion appear to shorten the time of onset of sero-conversion (Brunetto et al., 1991). It was found to be correlated with persistence viral detection in the blood circulation and increased ALT levels for at least 6 months after seroconversion (Brunetto et al., 1991). It was also found to be correlated with elevated frequency of cirrhosis development in patients (Brunetto et al., 1991). These show that the timing of the emergence of the mutation could have clinical implications on affected patients. Besides, Marusawa et al. (2000) found that the predominance of TAG mutation after seroconversion correlated with increased liver injury. The precore stop codon mutations were also associated with severe recurrent HBV infection after liver transplantation (Angus et al., 1995).</p>\r\n	<p>\r\n		&nbsp;&nbsp; This mutation has also been associated with increased susceptibility to coinfection by hepatitis D virus (HDV) (Raimondo et al., 1991). Hepatitis D virus is a satellite virus of HBV. This virus can only replicate in the hepatocytes that are infected by HBV. This is because HDV requires the proteins that are expressed by HBV in order to form its own nucleocapsid and has its own RNA genome packaged.</p>\r\n	<p>\r\n		&nbsp;&nbsp; Lindh et al. (1996) observed that carriers infected with the C1858 variants showed more liver damage than carriers infected with the T1858 variants. They suggested that carriers with the C1858 variants might remain longer in the HBeAg-positive phase or maintain some HBeAg production capabilities even after seroconversion to anti-HBe. They hypothesized that the HBeAg is a target for the necroinflam-matory process, thus causing more severe liver injury. However, patients infected with the C1858 variants responded better to interferon therapy than those infected with the T1858 variants (Zhang et al., 1996). In Malaysia, Ton et al. (2001) observed that many of the asymptomatic carriers and patients with HCC were infected with this T1858 variant than in those suffering from chronic hepatitis.</p>\r\n	<p>\r\n		&nbsp;&nbsp; Further, many studies found that simultaneous occurrences of the precore stop codon mutations and the core promoter mutations would result in more severe cases of liver injury (Brunetto et al., 1991; 1993; Laskus et al., 1998; Inoue et al., 1998; Honda et al., 1999).</p>\r\n	<p>\r\n		&nbsp;&nbsp; Although a lot of studies point toward the association of the above mutations with increased risks of liver injuries, many other studies did not find such correlations. Castro et al. (2001) demonstrated that the core promoter mutations were not linked to increased liver injuries. In the case of the C1858/ T1858 variants, no significant clinical differences were observed between patients infected by these two variants (Sakurai et al., 2004). Similarly, the issue of increased risk of liver injury associated with infection by HBV strains with simultaneous precore stop codon mutations and the core promoter mutations is not resolved yet as many other studies refute this claim (Okamoto et al., 1990; Hsu et al., 1995; Lindh et al., 1996; Pollicino et al., 1995).</p>\r\n	<p>\r\n		<strong>HBV Genotypes</strong></p>\r\n	<p>\r\n		&nbsp;&nbsp; The genotype system of HBV was first proposed by Okamoto et al. (1988) with an initial of 4 genotypic groups (i.e. A to D) classified, being distinguished based on an inter-genotypic divergence of 8.5 to 10.0 % between 18 complete HBV genomes and with a divergence of 1.1 to 2.7 % between isolates within the same genotype. The genotype classification was later extended to 6 genotypic groups (genotypes A to F), with addition of genotypes E and F, through phylogenetic analysis on 122</p>\r\n	<p>\r\n		sequences of the S gene by Norder et al. (1993). Genotype G was discovered by Stuyver et al. (2000) from HBV carriers in France and blood donors in the United States of America. They reported that this genotype has a minimum divergence of 11.7 % (genotype G versus E) and a maximum divergence of 15.3 % (genotype G versus genotype H) when compared with other genotypes over the whole genomes. This genotype is unique in that it has a nucleotide insertion of 36 bp, situated after the fifth nucleotide after the core translation initiation point (nucleotide 1901) (Stuyer et al., 2000). The latest addition to the family is the genotype H which was observed from HBV-infected patients in Central America (Arauz-Ruiz et al., 2002).</p>\r\n	<p>\r\n		<strong>Geographic distribution of HBV genotypes</strong></p>\r\n	<p>\r\n		The prevalence of the HBV genotypes can be localized into different geographic areas. Genotype A has been found to be predominant in Northern Europe and North</p>\r\n	<p>\r\n		Americawhile genotype D is predominantly found in the Mediterranean region (Magnius and Norder, 1995). In Asia, genotype A can be found in high frequency in India, Indonesia and the Philippines (Huy and Abe, 2004). Genotypes B and C are found predominantly in the Far East such as China, Japan and the South-East Asia region (Magnius and Norder, 1995). There is a tendency for genotype C to have higher prevalence in Northern mainland China and in Japan than genotype B (Orito et al., 2001). Genotype E is found mainly in parts of East, Central and West Africa. As for genotype F, it is found mainly in South and Central Americas as well as in some Pacific islands such as the Polynesia. So far, genotype G has been observed in the United States of America, Germany and France and could be found in Mexico as well (Stuyer et al., 2000; Vieth et al., 2002; Sanchez et al., 2002). As mentioned previously, genotype H seems to be localized to the Central America region.</p>\r\n</div>\r\n<div>\r\n	<p>\r\n		&nbsp;&nbsp; In the Malaysian context, a small study performed by Ong et al. (2005) through sequencing on the pre-S and S regions of the HBV genome extracted from sera of HBV infected Malaysian carriers revealed that the genotypes B and C were the most prevalent strains. This was confirmed in later study on a larger Malaysian population sample where the prevalence of genotype B was to be highest (56.9 %) followed by genotype C (31.4 %) (Lim et al., 2006).</p>\r\n	<p>\r\n		<strong>Co-infections by several genotypes</strong></p>\r\n	<p>\r\n		Besides single genotypic infections, simultaneous infections by two different HBV genotypic strains are possible. These events can occur in countries where endemicity of several genotypic strains of HBV is prevalent. One of the first evidence of co-infections by two different genotypes was reported by Kao et al. (2001). In their large study, the frequency of co-infections by genotypes B and C was found to be 0.8 %. Since then, other studies had revealed that co-infections could be quite common, with a rate of co-infections of between 4.4 to 17.5 % (Ding et al., 2003; Kato et al., 2003, Osiowy and Giles, 2003; Chen et al., 2004; Olinger et al., 2006). In the local setting, a study on Malaysian HBV carriers revealed that 7.8 % of the sample population studied was coinfected by genotypes B and C (Lim et al., 2006).</p>\r\n	<p>\r\n		&nbsp;&nbsp; Even more intriguing is the report of triple simultaneous infections by three different genotypic strains (Chen et al., 2004). The report stated that in a group of intravenous drug users infected by HBV, 0.9 % of them had triple infections by the genotypic strains of A, B and C.</p>\r\n	<p>\r\n		<strong>HBV genotypic subtypes</strong></p>\r\n	<p>\r\n		The main genotypes can be further divided into subtypes. These had been determined in a few studies based on phylogenetic analysis (Norder et al., 1994; Bowyer et al.,<br clear="all" />\r\n		1997; Bowyer and Sim, 2000; Mbayed et al., 2001). It was found that genotype C could be further subdivided into subtypes with genotype C <em>adrq </em>serotype being more common in the Pacific islands than in South-East Asia (Norder et al., 1993). Norder et al. (1994) showed that Cq- subtype of genotype C in the Pacific region also differed from subtypes found in the Asia region. A study by Lindh et al. (1997) using the PCR-RFLP on the S region showed that within genotype C, RFLP pattern C1 was present in all Northeast Asian carriers while South-East Asian carriers had another RFLP pattern, designated as C2. Furthermore, a later study has found that the genotype C can be subdivided into the subtypes Ce and Cs (Chan et al., 2005).</p>\r\n	<p>\r\n		&nbsp;&nbsp; A study performed on the urban population of South Africa by Bowyer et al. (1997) showed that there exists two subtypes for genotype A, designated as subtypes A and A&rsquo;. Mbayed et al. (2001) found the existence of four clusters of subtypes within genotype F which were associated with different geographical locations. Esteban et al. (1999) theorized that HBV genotypic subtypes were likely to evolve in different ethnic backgrounds over the centuries of HBV infections.</p>\r\n	<p>\r\n		&nbsp;&nbsp; In a study by Sugauchi et al. (2003), two distinct subtypes of genotype B were found, namely Bj and Ba. The main difference between these two subtypes is that the genome of Ba subtype has undergone recombination with the genotype C over the regions of the precore and core gene while in Bj, no such recombination occurred. In Japan, the Bj subtype is the dominant strain while Ba is found mainly in other Asian countries such as China, Hong Kong, Taiwan, Thailand and Vietnam.</p>\r\n	<p>\r\n		&nbsp;&nbsp; As more and more studies are conducted in this area, it is expected that more genotypic subtypes will be discovered. The consensus for distinguishing between genotypic subtypes now stands at 4% (Kramvis and Kew, 2005). A total of at least 24 genotypic subtypes have been identified so far (Schaefer, 2007).</p>\r\n	<p>\r\n		&nbsp;&nbsp; It remains to be established whether the HBV strains of genotypes B and C infecting Malaysians are of the subtypes Ba or Bj in the case for genotype B and of subtypes Ce or Cs for genotype C or it could be some other undiscovered subtype alto-gether.</p>\r\n	<p>\r\n		<strong>Recombinant hybrids of different genotypes</strong></p>\r\n	<p>\r\n		In certain situations, recombinations between HBV genomes of different genotypic strains were reported. As discussed earlier, one of this is the genotypic subtype Ba, which is a result of recombination between HBV genomes of genotypes B and C (Sugauchi et al., 2003). From the current literature, the majority of the recombinant hybrids observed are of genotypes B/ C and A/ D (Simmonds and Midgley, 2005; Yang et al., 2006). Other recombination hybrids that have been discovered are A/ B/ C, A/ C, A/ E, A/ G, C/ D, C/ F and C/ G (Schaefer, 2007). In some regions, the recombinant hybrids are the predominant HBV strain infecting the local population. In Tibet for example, it was observed that the recombinant hybrid of genotypes C/ D was the most prevalent strain detected (Cui et al., 2002; Wang et al, 2005; Zeng et al., 2005). Whether such hybrids exist among the Malaysian local HBV strains remain to be seen.</p>\r\n	<p>\r\n		&nbsp;&nbsp; From the various studies, it was observed that there is a tendency for the recombinatorial breakpoints to cluster at certain regions of the HBV genome. Some of these recombination breakpoints which commonly occur are at the preS1/ 2 region (nt 3150-100), the 3&rsquo;-end region of the Core gene (nucleotide 2330-2450) as well as at the 3&rsquo;-end region of the Surface gene (nt 650-830) (Simmonds and Midgley, 2005; Yang et al., 2006).</p>\r\n	<p>\r\n		&nbsp;&nbsp; However, the mechanism in which these events occur is still not clear. Some authors suggested that these could be due to recombinatorial events between the genomes of different genotypes (Hannoun et al., 2000; Owiredu et al., 2001). It is postulated that this phenomenon occurs in an environment when co-infections by several genotypes exists. HBV has a high replicative capability where up to 1013 viral particles can be produced everyday as well as high mutation rate due to the absence of proof-reading capability on the part of the viral polymerase (Nowak et al., 1996; Brechtbuehl et al., 2001). This leads to a proposal by Schaefer (2007) which states that the existence of such hybrids is a result of rapid adaptation of the HBV to new host immunological selections encountered in different human populations. Another more sophisticated theory on the occurrence of such hybrids is the theory of HBV genome mosaicism (Bowyer and Sim, 2000). This theory suggests that the existence of HBV genotypes are due to the modular construction of the HBV genome from different small segments of HBV genomes of various genotypes (Bowyer and Sim, 2000; Simmonds and Midgley, 2005). It is supported by a discovery of the activation or repression of the preCore-pregenomic promoter (enhancer II) by the transcription factor COUP-TF which is genotype-dependent (Fischer et al., 2006).</p>\r\n	<p>\r\n		<strong>The associations of HBV genotypes with clinical prognosis</strong></p>\r\n	<p>\r\n		Studies have shown that HBV genotypes correlated to different degree of severity of liver diseases. Kao et al. (2000a) observed that HBV genotype C associated more with severe liver disease while genotype B was associated with the development of HCC in young HBsAg-positive carriers. A further study revealed that patients with genotype C infections had slightly higher sera HBV DNA levels and significantly higher ALT levels than patients infected with genotype B (Kao et al., 2000b). Besides individuals infected with genotype B would undergo HBeAg seroconversion to anti-HBe at a younger age (Chu and Lok, 2002; Kao et al., 2002; Sumi et al., 2003).</p>\r\n</div>\r\n<div>\r\n	<p>\r\n		&nbsp;&nbsp; On the other hand, genotype A was found to be present in higher frequency in chronic hepatitis patients than in acute patients (Mayerat et al., 1999). In a study performed by Kobayashi et al. (2002a), they found that chronic hepatitis tend to occur more frequently in patients infected by genotype A than by genotypes B or C. Cases had also been reported where acute infections by HBV genotype A evolved more frequently into chronic infection (Kikuchi et al., 1998; Niitsuma et al., 1999). These findings could lead to the reasoning that in Western countries, where genotype A infections are prevalent, there is a high rate of chronic infection after HBV infection during adulthood (Sherlock et al., 1987; Heijtink et al., 1999; Lindh et al., 2000). Furthermore, Kobayashi et al. (2002b) showed that patients infected with HBV genotype A had higher HBeAg levels than &nbsp;those infected by genotypes B or C.</p>\r\n	<p>\r\n		&nbsp;&nbsp; In the Indian sub-continent, genotype D was more associated with severe forms of liver diseases than genotype A (Thakur et al., 2002) and that patients below the age of 40 years old had higher incidence of getting HCC when infected by genotype D than by genotype A. Mayerat et al. (1999) suggested that HBV genotype A has lower antigenicity than genotype D, thus it is less capable of inducing the immune clearance phase and so lead to a higher incidence of chronic hepatitis. In the Mediterranean region, infection by genotype D was associated with the prevalence of anti-HBe-positive chronic hepatitis (Chu et al., 2003).</p>\r\n	<p>\r\n		&nbsp;&nbsp; However, it is important to note that this issue is not firmly established yet. This is because some other studies did not find such correlations between the genotypes and the prognosis of the liver diseases (McMahon et al., 2001; Sumi et al., 2003). In Malaysia, it is not known whether such correlation exists as no study has been undertaken to look into this question.</p>\r\n	<p>\r\n		<strong>Correlations of HBV genotypes with mutations affecting HBeAg production and HBe/ anti-HBe status</strong></p>\r\n	<p>\r\n		Over the years of research on HBV genotypes, promoter mutation (A1762G1764&rarr;T1762A1764), precore stop codon mutations (TGG&rarr;TAG) and codon 15 variants (C1858/ T1858), various reports had been compiled on the correlations between these variables as well as the correlations between the genotypes with HBeAg/ anti-HBe status.</p>\r\n	<p>\r\n		&nbsp;&nbsp; The core promoter mutations (A1762G1764&rarr;T1762A1764) had been observed to be more common in patients with HBV genotype C infections than genotype B (Lindh et al., 1999; Kao et al., 2000b; Orito et al., 2001; Fang et al., 2002; Sakurai et al., 2004). However, in some studies, the correlations between HBV genotypes with core promoter mutations were not observed (Franca et al., 2004; Kidd-Ljunggren et al., 2004). This type of correlation was not observed in the Malaysian study undertaken by Lim et al. (2006) as well.</p>\r\n	<p>\r\n		&nbsp;&nbsp; HBV genotype A had been associated with C1858 variants (codon 15) (Li et al., 1993). Due to this, the cytosine (C) at nucleotide 1858 will not permit base-pairing to occur with adenine base (A) of nucleotide 1896 in genotype A. Thus, HBV genotype A has constraint in undergoing mutation from G to A at nucleotide 1896 to produce HBV strain with precore stop codon mutations, hence HBeAg-negative phenotype. This could partially explain for the high rate of chronic HBV infections in adult patients who are infected with this genotype, as observed in many European countries. This is also true in the case of genotype F (Kramvis and Kew, 1998). This is supported by the finding that patients infected with genotype A had higher HBeAg levels than those infected by genotypes B and C, as discussed above (Kobayashi et al., 2002b). Genotypes B, C, D and E had been associated with the T1858 variants (codon 15). However infrequently, the C1858 variants could still be observed among patients infected with genotypes C and D (Kramvis and Kew, 1998). This situation was observed in Malaysia where some C1858 variants were observed in some genotype C infected samples (Lim et al., 2006).</p>\r\n</div>\r\n<p>\r\n	&nbsp;&nbsp; Despite these, some HBV strains of genotype A could still undergo the TAG mutation (codon 28). This is possible when the strain develops the C to T mutation at nucleotide 1858, thus preventing the destabilization of the pregenomic RNA stem loop structure which is essential for viral replication (Li et al., 1993; Blackberg and Kidd-Ljunggren, 2000). Due to the tendency of genotypes B, C, D and E to be associated with the T1858 variants (codon 15), they are predisposed to TAG mutations (codon 28). Thus genotypes B, C, D and E were found to be associated more often with the TAG mutations (codon 28) than genotype A (Huy et al., 2004). However, some other studies did not observe such correlations. Orito et al. (2001) observed no significant difference between the predispositions to the precore stop codon mutation development between genotypes B and C. This phenomenon was not observed as well in a later study by Kidd-Ljunggren et al. (2004) on samples from various geographic origins. The suggested correlation between genotype B with precore stop codon mutations seems to contradict to a study by Lim et al. (2006) on Malaysian samples infected with HBV. Even though large numbers of individuals infected with HBV genotypes B were found in their study, no precore stop codon mutations were detected among them. This could imply that other factors might play a role here, such as the geographic origin of the HBV strains. However, this does not seem to be the case as other studies conducted on HBV-infected Malaysian samples did observe some precore stop codon mutants even though their genotypes were unknown (Ton et al., 1996; 2002; Yap et al., 2002).</p>\r\n<p>\r\n	&nbsp;&nbsp; Other than the associations of genotypes with the mutations, some studies had also observed the inequality in the distributions of HBeAg/ anti-HBe statuses between patients infected by different HBV genotypes. It was reported that patients infected by genotype B were more prone to be negative for HBeAg than in patients infected by genotypes C (Lindh et al., 1999; Sakurai et al., 2004). This was supported by a study on HBV-infected patients in Thailand (Sugauchi et al., 2002a). The reason behind these could be explained by the fact that patients infected by HBV genotype C experienced a longer delay in seroconversion to anti-HBe status (Lindh et al., 1999). Thus the consequence of this is that they will remain in the HBeAg positive status for a longer period of time. But this scenario was not observed among the Malaysian carriers infected with HBV genotypes B and C where no difference in the distributions of the HBeAg/anti-HBe statuses was observed (Lim et al., 2006). The collection timing of the HBV infected sera from the patients could be important with regards to the determination of HBeAg/ anti-HBe statuses of the patients as older patients tend to have seroconverted to anti-HBe.</p>\r\n<p>\r\n	<strong>Associations of genotypic subtypes with other variables</strong></p>\r\n<p>\r\n	It was observed by Sugauchi et al. (2003) that the Bj and Ba subtypes correlated differently with the core promoter mutations. They found that the Ba subtype was more associated with the core promoter mutations than with the Bj subtype (33 % vs. 15 %) while the precore stop codon mutations occurred more frequently in the Bj subtype (50 % vs. 18 %). Thus, the implication of these findings is that the correlation studies between genotypes and other factors such as disease prognosis or mutations associated with HBeAg production is not that straight forward anymore. This is because when correlation studies between genotypes and other variables were performed, the proportions of the subtypes of one particular genotype could significantly influence the final outcome of the results. The final correlation results might be dependent upon the varying degree of proportions of the subtypes since it was shown that the association between the subtypes and other variables could be different.</p>\r\n<p>\r\n	&nbsp;&nbsp; Besides, a few studies had found that genotypic subtypes are significantly correlated with the prognosis of the liver disease as well. Studies in Japan revealed that the subtype Ba was more associated with severe liver disease as well as had higher tendency to develop HCC than in patients infected with the subtype Bj (Sugauchi et al., 2002b; 2003; Orito et al., 2005). In Hawaii, it was observed that patients of Asian ethnicity (excluding Japanese) which were exclusively infected with the Ba subtype had higher rates of HBeAg positivity and core promoter mutations than the Bj subtype that was predominantly found to infect the Japanese (Orito et al. 2001; Sakurai et al., 2004). Another example of clinical difference resulted from different subtypes infections is shown in a study by Hayashi et al. (2007) in Japan where the genotypic subtypes of Ba and Cs were more frequently found in patients suffering from acute hepatitis B than in patients with chronic hepatitis B.</p>\r\n<p>\r\n	&nbsp;&nbsp; The varying proportions of the genotypic subtypes within the samples of studies could be the reason why contradictory results were reported when correlation studies between genotypes and other variables were performed. This includes a study carried out on a Malaysian sample population where no difference was ob-served between genotypes B and C and the core promoter mutations. This is in contrast to what has been observed elsewhere (Lim et al., 2006).</p>\r\n<p>\r\n	<strong>SUMMARY</strong></p>\r\n<p>\r\n	After many years of research on HBV, it is accepted that HBV is one of the causative agents for the development of acute and chronic hepatitis. Besides, infection by HBV is a risk factor for HCC. As the prevalence of HBV infection among the Malaysian population is relatively high, these infected individuals run the risk of developing the above conditions. Moreover a number of HBV variants exist which have diagnostic and clinical implications (Raimondo et al., 1991; Yotsumoto et al., 1992; Okamoto et al., 1994; Sato et al., 1995; Lindh et al., 1996; Kao et al., 2000a).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Mutations in the HBV genome can affect the expression of HBeAg. Examples of these are the core promoter dual mutations (A1762G1764&rarr;T1762A1764), the codon 15 variants (C1858/ T1858) and the precore stop codon mutations (TGG&rarr; TAG). The HBV can be categorized into eight genotypes (A to H) as well as genotypic subtypes and recombinants. These could differ in their affiliations with the mutations and with disease prognosis. Although vaccination program has been introduced into Malaysia, a substantial proportion of the Malaysian population is still afflicted with HBV infections though the prevalence has decreased. The number of studies undertaken in Malaysia on this virus is still considerably low. Therefore it is important that more studies on HBV in Malaysia are conducted so as to better understand the epidemiology, morpho-genesis and pathogenesis behavior of the virus in relation to the Malaysian po-pulation. One particular area of HBV research that is still lacking is the study on the genotypic subtypes which is getting more important as it has been shown to be possibly associated with other health risks.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(384, 381, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Coronary heart disease (CHD) is the major cause of morbidity and mortality in the world (American Heart Association, 2004). This trend has been predicted to continue until 2020 (WHO, 2005). In 2002, CHD caused 16.5 million deaths in the world and estimated to increase by 25 million annually (WHO, 2005). CHD is also the most common death in Malaysia and is a significant cause of disability among both men and women (Ministry of Health, 1998). It manifested as angina, silent ischemia, unstable angina, myocardial infarction, arrhythmias, heart failure and sudden death. Patients presenting with chest pain may be identified as having definite or possible angina from their history alone (Grech, 2004).</p>\r\n	<p>\r\n		Several studies have proven psychosocial factors attribute and link to the increment of CHD risks (Donald, 2001). The coping mechanism of adaptation to anxiety and depression among CHD varies (Kulik &amp; Mahler, 1993). An increase in anxiety among the CHD patients would augment the level of stress and this extreme stress level would then aggravate depression (Thompson &amp; Webster, 2004). The normal physiological responses to stress are related to an elevation of the heart rate, altered tissue perfusion, hypoxia and electrolyte imbalance. These physiological changes would increase demand for oxygen consumption which leads to ischemic chest pain, arrhythmia or sudden death among CHD patients (Cruickshank, Bradburg, &amp; Ashurst, 2000). According to Heikkla, Paunonen, Virtanen, &amp; Laippala, (1998), PCI is a common invasive diagnostic investigation that could result in high levels of stress among CHD patients.</p>\r\n	<p>\r\n		PCI was introduced in 1959 and it was considered as the gold standard for defining the anatomy of the main coronary arteries (Jowet &amp; Thompson, 2003). The only absolute way to evaluate an individual&rsquo;s cardiac status for CHD is to perform a PCI and to monitor the haemodynamic status.&nbsp; PCI can be safely performed as a day case procedure.&nbsp; The procedure takes about half an hour and is performed under local anesthesia (www.ijn.com).</p>\r\n</div>\r\n<p>\r\n	Anxiety is defined as &ldquo;the feeling of being very worried about something that may happen or may have happened, so that you think about it all time&rdquo; (www.longman.com/dictionaries). A per-son is conscious of the unpleasant emo-tional state of threat or danger which can affect one&rsquo;s behavior and physiological system (Leonard &amp; John, 2004). Several studies exploring the state of mind of PCI patients have been reported (Peterson 1991, Davis et al. 1994). There have been reports that PCI is a stressful and fearful procedure which could aggravate high levels of anxiety before and after the procedure. Finesilver (1980) reported that unrelieved anxiety usually increases the stimulation of sympathetic nervous system which lead to increment in cardiac workload. Increased sympathetic arousal has been reported to activate the occurrence of ventricular arrhythmias and sudden death (Leonard et al., 2004).</p>\r\n<p>\r\n	Depression is a normal phenomena expressed by the CHD patients, particularly if anxiety level is unrelieved and undetected (Lane and Mahler 2002, Jowett et al. 2003). Depression is defined as &ldquo;feeling of sadness that makes you think there is no hope for the future&rdquo; (www.longman.com/dictionaries).</p>\r\n<p>\r\n	Depression is expressed by a spectrum of mood disturbance ranging from mild to severe. The two cardinal symptoms are persistently pervasive, low mood and loss of interest or pleasure in usual activities (Leonard et al., 2004).</p>\r\n<p>\r\n	However, Marmot &amp; Stephen (2002) reported depressive illness and symptoms could have contributed to higher and stronger evidence for depression among CHD patients. It acts as an antecedent for CHD patients too. In addition, disability due to poor quality of life and depression are associated with higher healthcare costs. Hence, CHD patients may encounter increasing high risk of re-infarction, re-hospitalization, morbidity and mortality (Leonard et al., 2004).</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This research study is a cross sectional study conducted in three cardiology wards of the National Heart Institute (NHI) from January to March 2006. The objective of this study was to determine the incidence of anxiety and depression in patients before and after PCI. Primary data was collected using fully structured ques-tionnaires of the Hospital Anxiety and Depression Scale (HADS) adopted from Zigmond and Snaith (1983).&nbsp; HADS, is a 14-item questionnaire with fixed response statements (weight 0-3 for each item) divided into two separate sub-scales. The first sub-scales comprised of seven items to measure depression (HADS &ndash;D) and another 7 items to measure anxiety level (HADS- A).&nbsp; A score&nbsp; greater than 10 points&nbsp; equates to high significantly&nbsp; anxiety or depression level, 8-10 points is considered as moderate borderline and less than 8 points as low level. The same HADS questionnaires were used before and after PCI.</p>\r\n<p>\r\n	Probability sampling method was used, whereby all patients at NHI who fulfilled the inclusion criteria were recruited for this study. Sample size was calculated using the INFOPAC programme. 61 respondents participated in this study. The response rate was 100%. The inclusion criteria were patients who were scheduled for PCI (with or without angioplasty) and were able to understand Malay or English or both. The exclusion criteria includes critically ill patients (from coronary care unit), repeated angiogram due to complications after procedure such as pericardial effusion and cardiac tamponade and psychiatric pa-tients. There were two sets of question-naires; section A consisted of 14 items to measure anxiety and depression level HADS-D and HADS-A. Questionnaires were translated and back translated (English and Bahasa Malaysia). Section B consisted of 11 items on socio demo-graphic data of respondents using nominal and ratio scale. A similar set of question-naires were given to the patients before and after PCI.</p>\r\n<p>\r\n	Approval from the ethics committee (project code: FF-200-2005) of Faculty of Medicine, Universiti Kebangsaan Malaysia (UKM) and permission from the Chief Executive Officer (CEO) of NHI were obtained prior to commencement of the study. Socio demographic data were analyzed using the descriptive statistical analysis such as frequency and per-centages. The Pearson correlation-co-efficient test was used to determine the association between socio demographic data and anxiety and depression among PCI patients. One wayANOVA and t-test were also used to identify the relationship of respondents&rsquo; socio demography with anxiety and depression.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Respondents before and after PCI anxiety levels</em></p>\r\n<p>\r\n	Before PCI, 12 respondents (19.7%) reported the highest level of anxiety and reduction to 9 respondents (14.8%) after PCI. It was found that moderate level of anxiety were 9 (14.8%) respondents before PCI whereas there was an increase to 14 respondents (23%) after PCI respectively. More than half of the respondents showed low levels of anxiety, 40 respondents (65.6%) before PCI with reduction to 38 respondents (62.3%) after PCI.</p>\r\n<p>\r\n	<em>Respondents before and after PCI depression levels</em></p>\r\n<p>\r\n	Respondents before and after PCI depression levels were identified. There were seven respondents (11.5%) reported with high depression level before PCI which was reduced to one respondent (1.7%) after PCI respectively. Before PCI, seven respondents (11.5%) reported to have experienced moderate depression level with a slight increase to two respondents (3.3%) after PCI. The lowest level of depression scored the highest with 47 respondents (77%) before PCI and one respondent (1.7%) after PCI respondents.</p>\r\n<p>\r\n	<em>Respondents before and after PCI anxiety and depression levels.</em></p>\r\n<p>\r\n	There was a strong&nbsp; positive correlation between respondents before and after PCI anxiety levels (r =0.567, p value &lt; 0.05) and before and after PCI depression levels (r=0.526, p values &lt; 0.05).</p>\r\n<p>\r\n	<em>Respondents&rsquo; socio demographic data, before and after PCI with anxiety levels.</em></p>\r\n<p>\r\n	Respondents&rsquo; socio demographic data, before and after PCI with anxiety and depression levels were analyzed descriptively for frequency. The relationship between socio demographic data, before and after PCI anxiety and depression levels was analyzed using independent t-test and one way ANOVA.&nbsp; There were insignificant differences between respondents&rsquo; socio demographic data in relation with before and after PCI anxiety and depression levels.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The majority of respondents (before and after PCI) reported to have low levels of anxiety and depression in this study. Two thirds of the respondents showed a decrease in the level of anxiety and depression levels after PCI.&nbsp; This is consistent with the findings of Heikkla et al. (1998). At the NHI, it is mandatory for all patients and spouses to attend health</p>\r\n<p>\r\n	education such as &ldquo;pump talk&rdquo; and orientation carried out by the physician, nurses, cardiac angiographer and cardiac technician. These prerequisites knowledge could have assisted them in clarifying their doubts about PCI. Health education session may have contributed to low level of anxiety and depression. Other confounding factors such as well trained staffs, equipped facilities, conducive environment and adequate technology could have also be other influencing factors leading to low levels of anxiety and depression.</p>\r\n<p>\r\n	Results from this study showed an increase in their moderate levels of anxiety and depression after PCI among the respondents. It was found that most of the respondents attributed their feelings of nervousness and inability to relax to the laboratory environment which consisted of bulky machineries, health care providers with masks and gowned, and dim lighted room.&nbsp; Thompson et al. (2004), also reported that the noise of the X-Ray equipment, the semi darkness, the masked and gowned staff, and the movement of the table during positioning, aggravated the anxiety level of patients. In addition, they were anxious when informed of their progression and intervention in relation to the findings of PCI.&nbsp; According to Rice, Caldwell, Butler &amp; Robinson, (1986) it is better to inform the patients of pending PCI findings because the fear of anticipating the PCI findings can be greater than the procedure itself.</p>\r\n<p>\r\n	The levels of anxiety and depression in respondents before PCI were remarkably low; however, after PCI, their levels of anxiety and depression were even much lowered. Parker et al., (2006) reported that level of anxiety and uncertainties would be aggravated while waiting for revascu-larization.&nbsp; Meanwhile, Pederson et al., (2006) found only 41 (8%) respondents, out of 542 respondents gradually deve-loped significant depressive symptoms at 12<sup>th</sup> month post PCI.&nbsp; This result of the present study is similar with those previously published. It is hoped that future studies could include the follow up of post PCI respondents.</p>\r\n<p>\r\n	Results of this study showed a significant correlation between the presence of anxiety and depression both before and after PCI (p values, &lt; 0.05). This asso-ciation between anxiety and depression before and after PCI is congruent with the findings of Parker et al. (2006) who showed strong interdependence between an-xiety and depression among patients with acute coronary syndrome. Donald (2001) reported that depression and anxiety were prevalent among coronary heart disease patients.</p>\r\n<p>\r\n	However, there was no significant cor-relation between anxiety and depression levels to socio-demographic data. This could be due to the non inclusion of other factors that may contribute to anxiety and depression in this study. Sherry et al. (2005) stated that psychosocial factors were pivotal and greatly influenced psycho-logical processes of an individual during the phases of their coronary heart disease. According to Koivula et al. (2002), similar findings with regards to the anxiety level of coronary patients were interfaced with the patients&rsquo; social support and availability of resources.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, results of this study show that anxiety and depression levels before and after PCI was low. There was a reduction in the anxiety and depression levels after PCI but it was insignificant. The low level of anxiety and depression seen among the PCI patients at NHI could be due to the adequate facilities, orientation and health education provided to them before and after PCI by the physician, nurses, cardiac angiographer and cardiac technician. In addition, holistic psycholo-gical support from the support groups may have further assisted these patients in reducing the levels of anxiety and depression. This may help the PCI patients to cope to a better level and hence, lead to reduction in morbidity and mortality.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The development of this study would not have been possible without the contribution of many scholarly individuals. I sincerely would like to express my appreciation to Associate Professor Dr. Aliah Hanim M. Salleh, Encik Suhaimi Ahmad and Associate Professor Dr John Urban for their guidance and constructive critiques during various stages of development of this study.</p>\r\n<p>\r\n	Finally, I am further indebted to the Management of the National Heart Institute for granting permission to conduct this study.</p>\r\n', 1),
(385, 382, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Pregnancy is a time of growth and hope (Schroeder, 1996). Pregnancy is perceived by many pregnant women as a period of happiness in anticipation of motherhood.&nbsp; Women hope for a smooth journey in pregnancy without any complications and a normal fetal development. However, not all pregnant mothers experience joy and cheerfulness as some may experience anxiety, stress and depression. Anxiety is a dimension of stress (Brown, 2001) that occurs in response to internal or external stimuli and can result in physical, emotional, cognitive and behavioral symptoms (Relier, 2001). Depression, anxiety and stress are quite prevalent during pregnancy (Halbreich, 2005) and occurs in majority of the women with a prevalence rate of 30% (Matteson, 2001). Depression and anxiety, on the other hand, can cause a stressful pregnancy that can result in fetal distress, preterm delivery, low birth weight, postpartum disorders and other delivery complications.</p>\r\n<p>\r\n	&nbsp;&nbsp; Previous studies by Zigmond &amp; Snaith (1983) have shown that pregnant mothers experience a high anxiety and depression level for various reasons and is worse among high risk pregnant women. Several other studies have also shown that anxiety and depression during pregnancy may affect fetal development pregnancy outcome and. labour (Sjostrom et al, 2002; Mulder et al, 2002; Sandman et al, 1994). Literatures have also discussed that women who experiences anxiety disorders during pregnancy are at increased risk for intense postnatal depressive symptoms (Sandman et al, 1994, Chung et al, 2001).</p>\r\n<p>\r\n	&nbsp;&nbsp; Prevalence of anxiety disorders during pregnancy among the blacks was reported to be 40% (Barnett, 1996) and among Hispanic women 51% (Zayas et al, 2002). Prevalence was found to be high among women of low socioeconomic status (Seguin et al, 1995). The prevalence of anxiety and depression among high risk pregnant women in the local settings was never investigated and little is known about its prevalence and causes.</p>\r\n<p>\r\n	&nbsp;&nbsp; Iams (1998) reported that approximately one million women each year experience medical complications of pregnancy and are designated as at-risk or high risk. Of these, the majority are treated with bed rest in the hospital. Hospital environment compared to a home environment may cause some stress, anxiety and depression to them. Besides a hospital environment, there are many other factors that can contribute to the level of anxiety and depression among high risk pregnant women. Poor social support was found to have influenced the anxiety level of high risk pregnant women (Rodriguez et al, 2001). Biomedical factors such as medical condition, behaviour of ward staffs and unfriendly ward conditions could also influence the anxiety and depression level during their hospital stay. However, little is known about the contributing factors among high risk pregnant women in the local scenario and its negative effects during pregnancy.</p>\r\n<p>\r\n	&nbsp;&nbsp; The aim of this study was to determine the level of anxiety and depression among high risk pregnant women during their stay in the hospital and identify the factors influencing their level of anxiety and depression. Furthermore, there is a need to study the needs of high risk pregnant women during their hospital stay in HUKM as there has been no study done locally.</p>\r\n<p>\r\n	<strong>METHODOLOGY</strong></p>\r\n<p>\r\n	A descriptive cross-sectional study was conducted on 38 high risk pregnant women whose stay in hospital exceeded more than three days. The study was conducted in the obstetric ward of Hospital Universiti Kebangsaan Malaysia. High risk pregnant women whose stay is less than 3 days were excluded from the study. Pregnant women with placenta praevia, eclampsia, diabetes, hypertension were all considered as high risk in this study.</p>\r\n<p>\r\n	&nbsp;&nbsp; The instrument used here is a self-administered questionnaire which was divided into three (3) sections. Section A, B and C. Section A consisted of socio-demographic data, Section B consisted of the self-assessment questionnaire &ldquo;Hospital Anxiety Depression scale&rdquo; developed by Zigmund &amp; Snaith (1983) used to measure the level of anxiety and depression. Section C measured the factors that may influence their anxiety and depression level. Questionnaires that could contribute to the anxiety and depression among high risk pregnant women were prioritized by the researcher based on several literature search. Questionnaires were divided into five (5) headings: <em>information on</em> <em>disease</em>, <em>family matters</em>, <em>health service providers</em>, <em>ward conditions</em> and <em>finance</em>. A total of 18 questions related to the above headings were asked. The questionnaires were pilot-tested for its reliability and validity with a convenience sample of 10 high risk pregnant women. A panel of expert researchers reviewed the questionnaire and incorporated the changes to improve its clarity.&nbsp; Questions were prepared in two languages: Bahasa Malaysia and English and respondents could choose to answer in any of the preferred languages.</p>\r\n<p>\r\n	&nbsp;&nbsp; All data were coded and entered into SPSS (Statistical Package Social Sciences) for descriptive analysis. A three point Likert scale was used to measure the level of anxiety and depression. A score of between 0-7 was classified as having mild anxiety and depression and scores above 8 as having severe level of anxiety and depression. Factors influencing anxiety and depression were measured by frequencies and percentages.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<strong>Level of Anxiety and Depression</strong></p>\r\n<p>\r\n	Table 1 described the socio-demographic data of respondents. The majority of the respondents in this study were working mothers (71.1%) and 60.5% were in the second trimester. Their medical conditions vary from one another as 21.1% had hypertension, placenta praevia (18.4%) and (7.9%) diabetes. Of the 38 respondents, 22 (57.9%) were found to have experienced a severe level of anxiety and 16 (42.1%) was classified as having mild anxiety. Twenty-one (55.3%) high risk pregnant women were classified as having severe depression compared to 17 (44.7%) experiencing mild depression.</p>\r\n<p>\r\n	<strong>Factors that contribute to the level of anxiety and depression</strong></p>\r\n<p>\r\n	Out of the 38 respondents, 30 (78.9%) strongly identified &ldquo;<em>unsure of</em> <em>the length of stay in hospital&rdquo;</em> which is related to lack of information on disease as the most important contributing factor to their level of anxiety and depression. The other factors identified were in relation to family matters such as &ldquo;<em>being away from husband</em>&rdquo; (76.3%) and the <em>inability to care for her children</em> (76.3%). &nbsp;Twenty-four (63.2%) of the respondents&nbsp; identified &ldquo;<em>lack of information on disease</em>&rdquo; as a contributing factor to their level of anxiety and depression. The less important contributing factor to the level of anxiety and depression among the high risk pregnant women in this study were the &ldquo;<em>health service providers</em>. Respondents were satisfied with the service of health care providers as Table 3 shows the behaviour of support staffs, rough nursing care from nurses and rough treatment form doctors scored a low percentage (15.8%) which denotes its insignificance to the anxiety and depression level among high risk pregnant women. In relation to finance, 50 % <em>&ldquo;identified insufficient money to pay hospital bills&rdquo;</em> as a contributing factor to their anxiety and depression level.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	This study identified the level of anxiety and depression among high risk pregnant women during their hospital stay and the factors influencing their anxiety and depression. The findings of this study indicated that high risk pregnant women experienced a significant level of anxiety and depression during their hospital stay. The fact that anxiety and depression was detected in all the respondents (100%), is similar to the previous data and findings of Piyasil, (1998) and Raskin et al.(1990) whereby pregnant women have also shown depressive symptoms (38%) and anxiety symptoms (12%).&nbsp; Factors such as &ldquo;lack of information on disease&rdquo;, &ldquo;family matters&rdquo;, &ldquo;finance&rdquo; and &ldquo;ward conditions&rdquo; were all found to be the main contributing factors of their anxiety and depression. For all high risk cases, hospitalization is part of management recommended and many when agreed for admission were not told of the length of stay, thus, causing anxiety and depression to the pregnant women in this study. Since anxiety and depression during pregnancy was found to have resulted in poor pregnancy outcome (Rini et al, 1999; Hedegaard et al, 1996) including complications such as fetal distress, preterm delivery, low birth weight and other delivery complications, therefore, steps should be taken to critically evaluate the factors causing anxiety and depression during their hospital stay.&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp; Findings of this study could therefore, act as a reminder to all doctors that providing sufficient knowledge and information for high risk pregnant mothers concerning their condition, could help alleviate their anxiety. This is important as according to Lazarus &amp; Folkman, (1984), lack of knowledge and information could result in poor coping mechanism of the disease for the women and thus resulting in a stressful pregnancy. The functions of information and the number of benefits have also been clearly identified by many other researchers. Grahn &amp; Danielson (1996) and Ream &amp; Richardson (1996) reported that information does enable a patient to gain control over themselves as well as it contributes to patient&rsquo;s sense of control (Dennis, 1990). Patients with a strong sense of control not only improve the relationships between health care providers but also reduce the level of anxiety (Philip et al, 1990). Information also enables a patient to actively participate in their treatment thus improving patient compliance with treatment and also enabling them to prepare and plan for their future (Hinds et al, 1995).</p>\r\n<p>\r\n	&nbsp;&nbsp; Doctors should also be cautious and be made aware in using the label &ldquo;high risk&rdquo; on high risk pregnant women so as not to scare or upset them of their condition and only admit them to the ward when necessary. Majority of the women in this study are working women, therefore, worrying about &ldquo;being away from work&rdquo; will definitely cause some anxiety and depression among this high risk pregnant women. However, the importance of rest and treatment should be clearly emphasized to all high risk pregnant women to prevent any maternal and fetal complications.&nbsp; This finding also emphasizes the importance of patient education concerning their disease as this will enable high risk pregnant women to cope with their disease and pregnancy effectively.</p>\r\n<p>\r\n	&nbsp;&nbsp; Family matters were identified as another important contributing factor to the level of anxiety and depression of the pregnant women in this study. Women prefer to be at home and enjoy their pregnancy with family members as family support is important for a pregnant woman throughout her pregnancy. This finding is consistent with the findings of Maloni et al (2001) and Maloni &amp; Kutil (2000) as it was also found to be a major concern for women in previous studies (Maloni &amp; Ponder, 1997; Mercer &amp; Ferketich, 1988) whereby hospitalization was found to have disrupted family functioning and childcare.</p>\r\n<p>\r\n	&nbsp;&nbsp; Factors like &ldquo;ward condition&rdquo; is a modifiable factor and could be looked into by the hospital management to provide a friendly and home like environment thus suppressing the anxiety and depression incidences and improve the care of high risk pregnant mothers. A hospital environment is always different from a home environment. Both the designation of having a high risk pregnancy and treatment with hospitalization and bed rest could be a stimulus for developing negative affect or mood (Hammer &amp; Kenan, 1980; Kemp and Hatmaker, 1989; Maloni et al, 1993; Mercer &amp; Ferketich, 1988).</p>\r\n<p>\r\n	&nbsp;&nbsp; &ldquo;Health service providers&rdquo; were found to have performed their job well as respondents in this study were happy with support staffs, nurses and doctors attitude and behaviour as many did not identify them as a contributing factor to their level of anxiety and depression. On the other hand, finance was found to be a contributing factor to their level of anxiety and depression, And since finance can influence a patient&rsquo;s state of mind, therefore, they should be made aware of an &ldquo;easy financing scheme&rdquo; on discharge, so as to alleviate the fear of owing money to an institution. Maybe, the hospital administrator could consider an installment payment which could help to lessen the burden of some of the pregnant mothers.</p>\r\n<p>\r\n	&nbsp;&nbsp; Lastly, cognitive behavior psychotherapy aimed at assisting pregnant women to cope effectively with stressful situations could be applied on high risk pregnant women considering all pregnancies as precious. Cognitive behaviour psychotherapy has been reported to have improved psychological well-being and reduce poor pregnancy outcomes (Halbreich, 2005).</p>\r\n<p>\r\n	<strong>CONCLUSION </strong></p>\r\n<p>\r\n	High risk pregnant women in this study experienced a significant level of anxiety and depression and a number of influencing factors have also been identified. It is therefore important for nurses and doctors to be aware and sensitive to the influencing factors that could cause anxiety and depression to enable pregnant mothers to enjoy their pregnancy and childbirth. Hopefully, this will then reduce the &ldquo;anxiety&rdquo; and &ldquo;depression&rdquo; among high risk pregnant women during their stay in hospital. Nurses and doctors working in Obstetric &amp; Gynaecology wards should also attend continuous medical education (CME) and continuous nursing education (CNE) to update their knowledge on providing quality patient care. Organizers of CME and CNE should prioritize or emphasize more on the importance of information given to pregnant mothers on the issues identified in this study.&nbsp; An information tool known as the &ldquo;Clinical Information profile&rdquo; could also be developed as it will be useful in guiding nurses and doctors when providing information to high risk pregnant women during admission.</p>\r\n', 1),
(386, 383, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Primary dysmenorrhoea is defined as pelvic pain before and/or during menstruation in the absence of identifiable pelvic pathology (Dawood 1985). It affects about 90% of women worldwide and typically first presents in adolescents(Dawood 1985, Davis &amp; Westhoff 2001, Coco 1999).The incidence increases from early to late adolescents and becomes more prevalent in the middle and late adolescents when ovulatory cycles are well established (Greydanus 1997).</p>\r\n<p>\r\n	Primary dysmenorrhoea is believed to be associated with many factors including behavioural and psychological aspects. There is compelling evidence to indicate that increased production of prostaglandin will cause abnormal uterine activity resulting in uterine hypoxia and ischemia (Ylikorkala &amp; Dawood 1978). &nbsp;It has been shown that dysmenorrhoea among adolescents can adversely affect their personal life causing limitation of their social and academic performance (Klein 1998 &amp; Banikarim et al 2000).</p>\r\n<p>\r\n	&nbsp;&nbsp; In Malaysia, a study among university students showed that the prevalence of dysmenorrhoea was 58% and 40% within one year after menarche (Htut et al 1996). Other studies done worldwide showed that dysmenorrhoea was highly prevalent among adolescents ranging from 59.7% to 85% with more than 10% reported affecting school activities (Klein 1998).Several risk factors were identified to be associated with dysmenorrhoea, which includes early age of menarche, positive family history in mothers and sisters, and duration of menstrual flow (Kennedy 1997, Andersch &amp; Milsom 1982, Robinsonet al 1992).</p>\r\n<p>\r\n	&nbsp;&nbsp; There have been few published studies on dysmenorrhoea among adolescents in Malaysia but none from a rural society. Young girls from the rural area of Malaysia are more conservative compared to city girls. They usually suffer quietly. Furthermore, primary dysmenorrhoea can be easily alleviated by simple analgesics and it should not be the reason for them to miss school activities (Davis &amp; Westhoff 2001, Banikarimet al 2000). Hence, it is of utmost importance to determine the scale of the problem in rural districts, so as to enhance early diagnosis and proper management in order to reduce the negative social impact in the affected young girls. Finally with this study, it is the hope of the authors that the primary health care providers and the school authorities should be made aware of the problem.</p>\r\n<p>\r\n	The objectives of this study were to determine the prevalence of dys-menorrhoea among adolescent girls in a secondary school in Hulu Langat district, Selangor, to determine its association with the menstrual characteristics and its effect on school activities.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This was a cross-sectional study conducted at a public secondary school in Hulu Langat, Selangor from May to July 2003. A sample size of 300 female students was determined by a table developed by Krejcie and Morgan which was based on 90% certainty such that the sample and the population did not differ for more than 0.05 (Baumgartner &amp; Strong1998). Stratified random sampling was used to ensure all classes were included into the study. There were 1269 girls enrolled into secondary one to five classes for the year 2003, and 23.6% of students from each form were selected using random numbers table to account for the 300 sample size. They were selected according to the alphabetical order name lists given by the school teachers.</p>\r\n<p>\r\n	&nbsp;&nbsp; Girls who had not attained menarche or had been diagnosed with gynaecological problems were excluded. Informed consent was obtained from their parents and the selected students were gathered during their once-weekly physical education classes. During these sessions, the students were given a 20-item questionnaire which was collected immediately on completion.</p>\r\n<p>\r\n	&nbsp;&nbsp; The questionnaire included information on sociodemographic data, menstrual characteristics, intensity of pain, and school activities affected. School absence is defined as missing a school day and class absence is defined as missing individual classes. There were seven to eight classes taught per school day. A numerical rating pain scale was used to measure the severity of the dysmenor-rhoea. The pain score of 1 to 3 was considered as mild pain, 4 to 6 as moderate pain and 7 to 10 as severe pain. The questionnaire was pretested to ensure face validity.</p>\r\n<p>\r\n	&nbsp;&nbsp; The data was analyzed using SPSS statistic software version 11.0. Chi square test, student T test and analysis of variance were used for data analysis. A p-value of less than 0.05 with confidence interval of 95% was taken as statistical significant difference.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The girls&rsquo; age ranged from 12 to 17 years and they were divided into two categories; early adolescence (12 to 14 years) which made up 56.3% and middle adolescence (15 to 17 years) which was 43.7%. The majority were Malay girls (92%) and more than half of them came from families with a total income of less than RM 1000 per month (Table I). The mean age of menarche was 12.2 years and the mean duration of menstruation was 7.12 days with 63.7% of them having regular cycles.</p>\r\n<p>\r\n	&nbsp;&nbsp; The prevalence of dysmenorrhoea reported by these girls was 62.3% (n=187), with 71.8% of these from the middle adolescence group. The other reported common symptoms associated with menstruation were lethargy (54.3%) and headache (25.3%). Most of them experienced their first episode of dysmenorrhoea within one to two years of menarche.</p>\r\n<p>\r\n	&nbsp;&nbsp; Dysmenorrhoea was shown to be significantly higher in the middle adolescence group (p=0.003), girls with regular menstrual cycle (p=0.007) and positive family history in the first degree relatives (p=0.0001) (Table II). However, this study found no significant difference in the prevalence of dysmenorrhoea with the age of menarche (p=0.170) and the duration of menstruation (p=0.811) (Table II).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Negative effects on school activities reported by these adolescents included school absence, class absence, reduced concentration and inability to participate in sports activities. Analysis of variance or ANOVA (Table III) showed that those with severe pain (pain score 7 to 10) had significantly more school and class absences. This result was further enhanced by Pearson correlation test (Table III), which showed significantly increasing numbers of school and class absences with increasing pain score. Students with higher mean pain score also reported impaired concentration in class (p&lt;0.05) and inability to participate in sports activities (p&lt;0.05) (Table IV).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Prevalence of dysmenorrhoea found in this study was 62.3%. Although the prevalence rate of dysmenorrhoea varied in different studies, the current rate was considered high in a rural setting, as it was slightly higher than the prevalence (58%) found by Htut et al (1996) among university students in a Malaysian city one decade ago. The variation could be due to the differences in the method of collecting data, definition of variables, age distribution, ethnic proportions, cultural background and most importantly geographical location.</p>\r\n<p>\r\n	&nbsp;&nbsp; Dysmenorrhoea was significantly higher in the middle adolescence group and this was consistent with the study done by Klein (1998). This could be explained by the fact that primary dysmenorrhoea commonly occurs in ovulatory cycles which usually appears later after menarche. A regular menstrual cycle also signifies the establishment of ovulation, which explained the higher prevalence rate in those with regular menstrual cycles. Endometrial prostaglandin is believed to be the main aetiology in primary dysmenorrhoea which is elevated during ovulation (Dawood 1985, Davis &amp; Westhoff 2001, Ylikorkala &amp; Dawood 1978).</p>\r\n<p>\r\n	&nbsp;&nbsp; Family history of dysmenorrhoea in the first degree relatives had been attributed to the high prevalence of dysmenorrhoea. Apart from hereditary factor, a psychological aspect is also found to be the main contributor for this correlation (Stenchever 2001). Daughters may react to menstruation similarly like their mothers and they may share the same attitude and taboos towards menses.</p>\r\n<p>\r\n	&nbsp;&nbsp; Despite the fact that ovulation occurs later after menarche, this study showed no significant association between the preva-lence of dysmenorrhoea and the mean age of menarche. Even though other studies had found a significant association with the age of menarche, the populations studied were actually different. Ng et al (1992) had studied dysmenorrhoea in females with a wider age range (15 to 54 year old) whereas Andersch (1982) and Harlow (1996) had selected an older adolescent category (17 to 19 year old). This might have produced a different result. Moreover, primary dysmenorrhoea decreases with age and secondary dysmenorrhoea predominates in later life.</p>\r\n<p>\r\n	&nbsp;&nbsp; Dysmenorrhoea is known to cause physical and social disability resulting in school or class absence and inhibiting sports and school activities. The school and class absenteeism rate found in this study was almost similar to that reported by Banikarim et al (2000) with 38% school absence and 33% class absence. However, our result was much higher than reports made by Klein (1998): among African Americans and American whites which were 23.6% and 14% respectively. The variation in the school absenteeism rate and other activities affected in these studies could be related to the different cultures, ethnicity and variation in the pain threshold perception, which was a subjective matter. Furthermore, other studies used different pain scales, which may be difficult to compare with one another. Adolescent girls with severe dysmenorrhoea were significant positively correlated to school and class absenteeism. With significant absenteeism, one important outcome to measure is the academic performance of these girls. However there are many confounders, thus it was not feasible in this study.</p>\r\n<p>\r\n	&nbsp;&nbsp; Besides absenteeism, the students also reported that dysmenorrhoea affected their concentration in class and participation in sports activities. The level of pain corre-lated positively with the limitation of their school activities. Since dysmenorrhoea had negative effect among adolescent girls, school teachers and health care providers should have better awareness regarding this problem as it can be easily treated with simple analgesics.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Prevalence of dysmenorrhoea was high among adolescent girls in this rural school in Selangor, Malaysia. It was significantly more among students in the middle adolescence (14 to 17 years old), with regular menstrual cycles and positive family history. Girls with more severe dysmenorrhoeic pain had significant effects on their school activities. Awareness should be brought to the local school health authority and the primary health care providers to actively identify this problem and manage appropriately. Future study need to explore the adolescents&rsquo; perception and help-seeking behaviour towards primary dysmenorrhoea to guide health care providers in planning a more effective and strategic screening program.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(387, 384, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n	<p>\r\n		Female sexual functioning is a state of ability to achieve sexual arousal, lubrication, orgasm and satisfaction and results in wellbeing and state of wellness, with good quality of life (Briana, 2001). One way to assess sexual activity among women is to determine the frequency of sexual intercourse (SI). Female sexual dysfunction (FSD) is a state of disorder in any of the above sexual domains. The Diagnostic and Statistical Manual of Mental Disorders (DSM-IV) (American Psychiatric Association, 1994) has laid down strict criteria for FSD - it requires that the interpersonal distress must have occurred for at least six months. According to Salonia et al. (2004), both sexual functioning and FSD is a multifactorial condition with anatomical, physiological, medical, psychological and social components. Cultural and religious factors also have great impact on human sexual profiles (Lightner 2002).&nbsp; FSD includes some modifications to the DSM-IV system. This system classifies the following disorders: (1). Sexual Desire Disorder, which is further classified into: a. Hypoactive Sexual Desire Disorder, and b. Sexual Aversion Disorder, (2). Sexual Arousal Disorder, (3). Orgasmic Disorder and (4). Sexual Pain Disorder.</p>\r\n	<p>\r\n		Hypoactive sexual desire disorder is a persistent or recurrent deficiency (or absence) of sexual&nbsp; feelings or desire for or receptivity to sexual activity, which causes personal distress (Basson et al 2000) and&nbsp; affects women more frequently than men (Berman et al 2001). Sexual Aversion Disorderis a persistent or recurrent phobic aversion to and avoidance of sexual contact with a sexual partner, which causes personal distress (Basson et al 2000) and it could be associated with past sexual trauma or sexual abuse (Rosen 2000). Sexual arousal disorder is a persistent and recurrent inability to attain or maintain sufficient sexual excitement, which in turn causes personal distress. Basson et al 2000 reported lack of subjective excitement, often manifested as a lack of vaginal or other somatic response with sexual stimulation. Women with problems of sexual arousal can have a purely psychological component (Schnarch 1997; Kaplan 2002) or medical problems (Kaplan 2002).&nbsp; Studies of the general population and sex therapy clinic populations indicate that the prevalence of female orgasmic disorder ranges from 24% to 37% (Rosen 2000). Certain drugs such as antipsychotic and antidepressant also have negative impact on sexual function (Smith 2002; Nurnberg 2003). Dyspareunia is a recurrent or persistent genital pain associated with sexual intercourse (Basson et al 2000) and has both physical and psychological components &ndash; contributed by several health conditions (eg. diabetes mellitus and pelvic pathologies) or psycho-logical distress and hostility (Nusbaum 2003; Schnarch 1997). Vaginismus, the motor arm of sexual pain disorder, is a recurrent and persistent involuntary spasm of the musculature of the outer third of the vagina that interferes with vaginal penetration and causes personal distress (Basson et al 2000; Spector &amp; Carey 1990).</p>\r\n</div>\r\n<p>\r\n	As sexual issues are still relatively considered a taboo in Malaysia and are&nbsp; not usually&nbsp; discussed openly in such multi-ethnic society (Hatta 2006), the under-standing of sexual problems is important as it would help physicians to understand issues in everyday human life. Sexual profiles and functioning in Malaysian women can be used as a guide for clinicians to understand the magnitude of female sexual difficulties in our community, so that help can be offered to them. The objective of this study was to compare sexual functioning among Malaysian women in a primary care setting between those who had low and high frequency of sexual intercourse.</p>\r\n<p>\r\n	<strong>SUBJECTS AND METHODS</strong></p>\r\n<p>\r\n	This was a cross-sectional study on women attending a primary health clinic. It was conducted over a period of four months (March to June 2005) at one of the government primary health care clinics located in Bandar Tun Razak, a rather busy suburban area of Kuala Lumpur. This study used a non-probability sampling (universal sampling) method. <strong>Inclusion criteria</strong> included: (i) female subjects; (ii) aged between 18 and 70 years old; (iii) married and have a sexually active partner; (iv) ability to read and understand the study languages (Malay or English); (v) consent for participation in the study. <strong>Exclusion criteria </strong>included: (i) chronic and severe medical illness/illnesses; (ii) psychiatric illness/illnesses; (iii) pregnancy; (iv) post-partum period of two months or less.&nbsp; The instruments used in this study were: 1. Sociodemographic and Marital Profile Form; 2. The Malay Version of Female Sexual Function Index (MVFSFI); 3. The Mini International Neuropsychiatric Inter-view (MINI).</p>\r\n<p>\r\n	<em>1. &nbsp;&nbsp; &nbsp;Sociodemographic and Marital Profile Form</em><br />\r\n	<br />\r\n	This brief questionnaire was devised to obtain respondents&rsquo; sociodemographic and marital information. It included the name, age, educational level, past medical history, employment status, monthly family income, menstrual history of the respondents, duration of marriage, age of the respondents&rsquo; husband, number of children and frequency of sexual intercourse.<br />\r\n	<br />\r\n	<em> 2.&nbsp;&nbsp; &nbsp;Malay Version of Female Sexual Function Index (MVFSFI)</em><br />\r\n	<br />\r\n	Sexual dissatisfaction was measured in this study using the sexual satisfaction domain of the Malay Version of the Female Sexual Functioning Index (MVFSFI). The original Female Sexual Function Index (FSFI) was developed by Dr. Raymond Rosen. It is a 19-item, multidimensional self-report measure of female sexual functioning. It covers six basic domains of female sexual functioning: desire, arousal, lubrication, orgasm, satisfaction, and pain (Rosen 2000). It is a brief, multidimensional self-report measure of sexual functioning that has been validated on a clinically diagnosed sample of women with female sexual arousal disorder (FSAD) (see table 1). Each domain has two to four questions with five to six options from which the patient chooses the one that most likely indicates her sexual function during the four weeks prior to the day they were given the questionnaire. The FSFI was translated into Bahasa Malaysia (BM) and validated in 2005 in Malaysia with the permission of Dr. Rosen (Norni and Hatta, 2005). The reliability test for agreement using the Pearson product-moment correlation co-efficient (r), ranged from 0.767 to 0.973. Internal consistency using Cronbach&rsquo;s alpha ranged from 0.87 to 0.97. A total score of &lt; 55 was used as the cut-off point for MVFSFI to distinguish between women with sexual dysfunction and those without (with a sensitivity of 99% and specificity of 97%). The lower the scores, the higher the women would suffer from FSD (Norni and Hatta 2005). Based on the validity study of MVFSI, the cut-off score is &lt; 9 for sexual arousal disorder (sensitivity 77% and specificity 95% ), &lt; 10 for disorder of lubrication (sensitivity 79% and specificity&nbsp; 87% ), &lt; 4 for orgasmic disorder (sensitivity 83% and specificity&nbsp; 85% ), &lt; 11 for sexual dissatisfaction (sensitivity 83% and specificity&nbsp; 85% ), and &lt; 7 for sexual pain disorder (sensitivity 86% and specificity&nbsp; 95% ).<br />\r\n	<br />\r\n	<em> Mini International Neuropsychiatric Inter-view (MINI)</em><br />\r\n	<br />\r\n	The MINI was used to exclude any respondents with psychiatric illness from this study. This is a brief structured interview for major Axis I psychiatric disorders in DSM-IV and ICD-10. The inter-rater reliability for this study was ascertained by administering the instrument on 10 cases selected randomly. This was done by two of the authors and yielded a kappa value of 1.</p>\r\n<p>\r\n	Approval was obtained to conduct the study from the university research ethics committee as well as from the administration authority of the particular clinic. All respondents who fulfilled the inclusion criteria were given an explanation about the study. A written consent was obtained from them. They were assured with regards to their anonymity and the confidentiality of the data obtained. A coding system was used to identify the respondents if it was necessary. After the MVFSFI was completed, each respondent was engaged in a clinical interview for diagnosing sexual dysfunction based on the DSM-IV criteria (American Psychiatric Association 1994) and administered the MINI for exclusion of the other psychiatric illnesses.</p>\r\n<p>\r\n	Analysis of the data was done using the computer program, Statistical Package for Social Studies (SPSS) version 11.5. The relationship between the study parameters was analysed using appropriate statistical tests. The chi-square test (&chi;&sup2;- test) was used to compare sexual functioning between the low and high sexual frequency groups of women.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Two hundred and forty eight patients who attended the Bandar Tun Razak primary care clinic, Cheras, Kuala Lumpur were invited to participate in the study. However, 18 patients were unable to complete the study because they were unable to make time (6 patients), did not feel comfortable</p>\r\n<p>\r\n	with the questions (7 patients) and did not bring their reading glasses to the clinic (5 patients). The response rate was 93% (230 subjects). Two patients who were screened and diagnosed to have anxiety disorder and major depressive disorder respectively by MINI were excluded. The demographic data of the respondents involved in this study are shown in table 2.</p>\r\n<p>\r\n	Almost half of the women (44.3%) in the sample studied had sex 1 - 2 times a week (figure 1). Another 42.4% had sex 1 - 2 times a month or less, and 13.5% had sex 3 - 4 times or more a week. Women who had SI 3 - 4 times a week or more was arbitrarily considered as having a high frequency of sexual intercourse (High SI) and women who had SI 1-2 times a week as having low frequency of sexual intercouse (Low SI).</p>\r\n<p>\r\n	Women above 45 years of age, married to an older husband and with a low frequency of sexual intercourse (Low SI), were more likely to suffer from sexual dysfunction, (FSD) compared to those with younger age, married to a younger husband and with a high frequency sexual intercourse (High SI).</p>\r\n<p>\r\n	Women who were less sexually active (SI &le; 1 &ndash; 2 times per week) were found to be less sexually aroused (&chi;&sup2;= 25.9, p&lt; 0.001), less orgasmic (&chi;&sup2;=19.8, p&lt; 0.001), less lubricated during sexual activity (&chi;&sup2;=11.1, p&lt; 0.001), experienced sexual pain (&chi;&sup2;=4.3, p = 0.033) and were less satisfied sexually (&chi;&sup2;=12.6, p&lt; 0.001).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Satisfactory sexual activity is an important element for human well-being, for better quality of life and motivation (Schnarch 1997). Sexual activity, such as the frequency of sexual intercourse can be a barometer to assess sexual functioning profiles (Hatta 2006). This study was an attempt to compare sexual functioning profiles between women with low and high frequency of SI and the risk of FSD.&nbsp; Before this, the issue of FSD was only disclosed to the traditional healers or between spouses in the bedroom, neglected and remained untreated (Hatta 2006).</p>\r\n<p>\r\n	Malaysian women in our primary care setting population were relatively young with a high level of educational back-ground. Their monthly family income was fair (40% had incomes ranging from RM 1,000 to RM 1,999) with more than half of the women being married for more than 10 years. Nearly half of them were very active sexually, having SI <u>&gt;</u> 1 &ndash; 2 times per week, slightly more than half were from the pre-menopausal age group. The prevalence of FSD among the local urban Malaysian population in this primary care setting was 29.6%.&nbsp; Research carried out in the west found that low sexual activity was associated with more sexual dysfunction (Laumann et al 1999).&nbsp;&nbsp; This was also shown in multi-centre studies in Asia (Nicolosi et al 2005). However, in the Nicolosi study, the mean age of their population was slightly older, hence the subjects were more likely to suffer from medical and gynaecological problems compared with the present study.&nbsp; The average range of sexual activity, as reflected by frequency of SI 1 &ndash; 2 times per week in the majority of our population is</p>\r\n<p>\r\n	almost similar with other studies in Western population (Nicolosi et al, 2005). Although sexual activity can be reflected by other behaviour like kissing, holding hands and sexual fantasy, the frequency of SI is considered one of the ways to determine their sexual performance.</p>\r\n<p>\r\n	Based on this research, Malaysian women with a low frequency of SI reported more sexual dysfunction. Their sexual function in all domains of arousal, lubrication, pain, orgasm and satisfaction were affected. The use of a validated questionnaire is an important effort to look at the magnitude of sexual problems (Meston 2003) in women with a low and high frequency of SI.&nbsp; However, we are not sure whether this association is causative or consecutive, but these findings are alarming enough to mobilise clinicians to address FSD in these group of women.&nbsp; It appears that sexual dysfunction in this study has affected those women who were older and who were logically married to older husbands. A woman&rsquo;s sexual functioning may be affected by psycho-logical and biological factors (Bachmann et al 2002; Berman et al 2001) and women with lack of sexual activity may have FSD due to negative perception on sex (Kaplan 2002). Sexual dysfunctions in older women may also be contributed by the factors in their husbands who are presumably also older and in whom sexual dysfunctions are more common which may be partly due to age-related medical problems.</p>\r\n<p>\r\n	In many situations, addressing female sexual functioning and FSD is more difficult than male sexual dysfunction (Nicolosi et al 2005), partly because sexual function in women is more complex and does not follow the linear male sexual response cycle (Whipple 2002). Dr Whipple high-lighted the difficulty in studying FSD, where so many non-anatomic and non-physio-logical factors come into play. Male sexual dysfunction can be more objectively defined and diagnosed, and interventions can be more objectively ranked with regards efficacy compared to FSD (Baumeister et al 2001). Furthermore, SI frequency, a measure used for male sexual function, cannot be used as an accurate marker of female sexual function because women may still be able to remain sexually active with their partner while experiencing FSD (McHorney 2004). Female sexual function may also be more dynamic than male sexual function. Although there are significant anatomic and embryologic parallels between men and women, the complex nature of FSD is clearly distinct from that of the male.</p>\r\n<p>\r\n	Our study was unique because a validated Malay psychometric instrument to assess female sexual function was used whereas other studies (eg. Nicolosi et al 2005) did not use validated questionnaires. However, there were a few limitations of this study.&nbsp; Firstly, our samples were drawn from a primary care population and did not reflect the true community population. Secondly, we did not analyse the sexual functioning of the husbands of the women in our study. Female sexual dysfunction was associated with male sexual dys-function, especially erectile dysfunction, ED (Selahi et al 2004). The research was conducted in an urban government primary care clinic which was busy and over crowded.&nbsp; Patients may be reluctant to ask questions during evaluation, despite a high prevalence rate of FSD. A study by Nusbaum et al (2000), demonstrated the prevalence of sexual concerns in women seeking routine gynecologic care. Stevenson (2003) commented, this might be due to the perception or the actuality that the physician is too busy, or he or she is not approachable. Similarly, most health care providers fail to address sexual history as a part of the medical history (Stevenson 2003). In terms of respondents, this study only included married women with a sexually functioning partner. Those who were not married (single, divorced or widowed) were excluded from this study because in Malaysia, the society at large is unable to accept extramarital sexual relationships. However, many unmarried Malaysian women are sexually active (Personal communication: Ismail Thambi 2005).</p>\r\n', 1),
(388, 385, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n</div>\r\n<div>\r\n	<p>\r\n		The incidence of thyroid nodules among adult population is between 4-7%, however, less than 5% of these nodules are malignant (Sclabas et al 2003). Few people die from thyroid carcinoma, on the other hand, thyroid surgery is associated with a number of risks like recurrent laryngeal nerve injury, hypoparathyroidism etc.&nbsp; Therefore, there is a need for an accurate test to screen the nodules that are likely to harbour malignancy and thus require operation while avoiding surgery of benign nodules.&nbsp;</p>\r\n	<p>\r\n		Fine needle aspiration cytology was first described in the 1930s by Martin and Ellis, however it became widely accepted in the United States only in the late 1970s (Leonard et al 1997, Matesa et al 2002). Currently, fine needle aspiration (FNA) has emerged as one of the first-line diagnostic techniques in the evaluation of thyroid nodules. It is a simple, cost-effective and safe procedure with few complications. The main purpose of thyroid FNA is to select nodules that require surgery from those that do not. It can also be diagnostic for certain lesions such as classic nodular goiter, Hashimoto&rsquo;s thyroiditis, papillary carcinoma, medullary carcinoma, ana-plastic carcinoma and metastatic carci-noma (Matesa et al 2002).</p>\r\n	<p>\r\n		The aim of this study is to determine the diagnostic accuracy of FNA as the initial diagnostic modality in the evaluation of a thyroid nodule.</p>\r\n	<p>\r\n		<strong>MATERIALS AND METHOD</strong></p>\r\n	<p>\r\n		A total of 2131 FNA thyroid were performed from January 1995 till December 2000 in Hospital Universiti Kebangsaan Malaysia (HUKM), a teaching hospital in Kuala Lumpur. Out of these, 441 FNAs were unsatisfactory for cytological evaluation. Of the remaining 1690 satis-factory samples, histopathological diagnoses were available for 361 cases.</p>\r\n	<p>\r\n		Fine needle aspirations of palpable masses were performed by Pathology Registrars and Cytopathologists. Deep-seated lesions were performed by radiologists using ultrasound-guidance. The aspiration technique was the standard one described in the literature (Orell et al 2002). The thyroid mass was examined clinically to note its mobility, consistency and size. Cervical lymph nodes were palpated to detect enlargement. The skin was sterilized with alcohol. No local anaesthesia was used. Aspiration was performed under negative pressure using a disposable 23-gauge needle attached to a 20 ml syringe that was fitted to a Cameco syringe holder. Direct smears were prepared from the aspirated material. The procedure was repeated two to three times at different areas of the thyroid mass. In cases of cystic lesions, the cyst content was evacuated and smears were prepared from the cyst fluid. Reaspiration was performed if there was a remaining solid mass palpable.</p>\r\n	<p>\r\n		At least two smears were fixed with alcohol and two air-dried. Alcohol-fixed smears were stained by the Papanicolaou method while the air-dried smears were stained by the May-Grunwald-Giemsa technique. All the smears were screened and reported by the Registrars undergoing cytopathology training and reviewed by the Cyto-pathologist on-call. The cytologic smears were interpreted according to the criteria described by Orell (Orell et al 2002), Ramzy (Ramzy 2001) and Cibas (Cibas and Ducatman 2003).</p>\r\n</div>\r\n<p>\r\n	Smears which contained no diagnostic cellular material or were heavily blood-stained are considered unsatisfactory for cytological evaluation. A repeat FNA was recommended in these cases.</p>\r\n<p>\r\n	The cytologic diagnosis was classified as benign (goitre, Graves disease, colloid cyst, and Hashimoto&rsquo;s thyroiditis), incon-clusive (follicular neoplasms and Hurthle cell tumours) and malignant (papillary carcinoma, medullary carcinoma, ana-plastic carcinoma, squamous cell carci-noma and non-Hodgkin lymphoma).</p>\r\n<p>\r\n	Subsequent to FNA, the histopatho-logical reports of the cases that were operated on were traced from the histo-pathology files. The final histopathological diagnosis was then compared with the corresponding cytological diagnosis and cyto-histopathological correlations were established in 361 cases.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	Cases with cytological diagnosis of follicular neoplasms and Hurthle cell tumours were regarded as inconclusive because it is not possible to differentiate between follicular adenoma (benign) from follicular carcinoma (malignant) based on cytological assessment. Hence, these cases were not included in the calculation for sensitivity, specificity, positive and negative predictive values and diagnostic accuracy. Unsatisfactory cases were also excluded from calculation.</p>\r\n<p>\r\n	True positives (TP) were defined as cases diagnosed as malignant on cytology which were histologically confirmed. False positives (FP) were those diagnosed as malignant on cytology that were benign on histology. True negatives (TN) were benign on both cytology and histology. False negatives (FN) were negative on cytology but positive for malignancy on histology. The diagnostic accuracy was calculated as: (TP + TN) / (TP +FP + TN + FN)</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	There were 2131 FNA thyroid performed in HUKM from January 1995 till December 2000. Out of these, 441 (20.7%) were reported as unsatisfactory,&nbsp;&nbsp; thus leaving 1690 (79.3%) satisfactory cases for cyto-logical evaluation. Histopathological diag-noses were available for 361 cases.&nbsp; Correlation between cytological and histo-pathological diagnosis was done on these cases.</p>\r\n<p>\r\n	From the 361 cases, 253 (70.1%) were classified as benign, 61(16.9%) malignant and 47 (13.0%) were inconclusive on cytology. These results are shown in Table 1.</p>\r\n<p>\r\n	The detail of cytological and the corresponding histopathological diagnosis is shown in Table 2. Histopathological examination confirmed the benign lesions in 245 (96.8%) of the 253 cytologically benign cases, which represent true negative cases. In these cases, FNA cytology correctly diagnosed 216 (92.7%) of the 233 goitres. There were 18 cases diagnosed as cyst on cytology. Out of these, subsequent histology revealed eight cysts, five goitres, two Hashimoto&rsquo;s thyroiditis, one Hurthle cell tumour, one follicular adenoma and one papillary carcinoma.&nbsp;</p>\r\n<p>\r\n	There were eight (3.2%) false negative cases which included six papillary carci-noma cytologically diagnosed as goitre, one papillary carcinoma diagnosed as cyst and one follicular carcinoma which was interpreted as goitre on cytology.</p>\r\n<p>\r\n	Of the 61 patients diagnosed as malig-nant on&nbsp; FNA cytology,&nbsp; 57 (93.4%)&nbsp; were confirmed malignant on histopathological examination, thus were true positives (41 papillary carcinoma, one follicular carcinoma, one medullary carcinoma, 11 ana-plastic carcinoma, one squamous cell carcinoma and one non-Hodgkin lym-phoma). There were&nbsp;&nbsp; four (6.6%) false positive cases. These cases were cytologically diagnosed as papillary carcinoma from which three were histologically diag-nosed as goitre and one as follicular ade-noma.Table 2 depicts the detail of cytological and the corresponding histopathological diagnosis. Table 3 summarises the cytological and corresponding histopatho-logical diagnosis.</p>\r\n<p>\r\n	We classified 47 cases as inconclusive (41 follicular neoplasms and six Hurthle cell tumours) because it is not possible to differentiate between follicular adenoma and follicular carcinoma based on cytological assessment alone. The same criteria applies for Hurthle cell adenoma and carcinoma. Of the 41 follicular neoplasms, six were malignant on histo-logy (five follicular carcinoma and one papillary carcinoma) while 31 were confirmed follicular adenoma and four goitres. Two of the six Hurthle cell tumours diagnosed on cytology turned out to be Hashimoto&rsquo;s thyroiditis on subsequent histopathological examination, and in the other four, the diagnosis of Hurthle cell tumour was correctly made.</p>\r\n<p>\r\n	From the data, our sensitivity rate for thyroid FNA is 87.7% and our specificity rate is 98.4%. Our positive predictive value is 93.4% and our negative predictive value is 96.8%. The diagnostic accuracy of FNA thyroid at our center is 96.2%. The formula used and the calculations are shown in Table 4.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	For thyroid FNA cytology to be clinically useful, a satisfactory sample must be obtained. Smears from thyroid aspirates are considered satisfactory when the material is representative of the lesion, adequate in quantity and the cyto-preparation is excellent. Our unsatisfactory rate is 20.7% which is on the high side compared with other previous studies which reported unsatisfactory rates between 0 to 25% (Ramachandra et al 1995).&nbsp; This could be attributed to the aspirations being performed by Trainee Registrars, who had limited experience during the initial part of their training. The quality of the smears usually improved as each student completed his or her three months cytology training. At our centre, a repeat FNA will be done for cases classified as inadequate. According to the Papanicolaou Society of Cytopathology Task Force, an acceptable rate of inade-quate smears should be kept less than 15% (The Papanicolaou Society of Cyto-pathology Task Force 1996).</p>\r\n<p>\r\n	In this study, we obtained a sensitivity rate of 87.7% and a specificity rate of 98.4%, which gives a diagnostic accuracy of 96.2%. Our results are comparable with several other studies which reported sensitivity rates between 65% to 98%, specificity rates between 72% to 100% (Sanggali et al 2006) and diagnostic accuracy rates between 70% to 90% (Vojvodich et al. 1994). The sensitivity, specificity and diagnostic accuracy depend on the experience of the person performing the aspiration and the cytopathologist interpreting the smear, the statistical method employed and whether the folli-cular lesions were included or not; and if they were included whether they were classified as benign or malignant. In our analysis, we did not include the cases classified as&nbsp; inconclusive which represent follicular neoplasms and Hurthle cell tumours, because the objective of this study is to calculate the diagnostic accuracy of fine needle aspiration cytology in thyroid lesions at our centre rather than its impact on clinical management.</p>\r\n<p>\r\n	Our false negative rate is 3.2%. This figure is in agreement with the other studies which reported false negative rates between 1-11% (Sanggali et al 2006). False negative rate is a concern because a missed malignant lesion will result in dire consequences for the patient. Review of these cases showed that most of them were due to sampling errors especially in the case of a cystic lesion where papillary carcinoma was missed. One case of follicular carcinoma was cytologically diag-nosed as goitre (Table 2). This was also considered as sampling error because the relative abundance of cellular material lacking in microfollicular pattern in a background of moderate amount of colloid as seen in this case would inevitably render a cytologic diagnosis of hyperplastic goitre. After all, one has to make diagnosis based on the available cytologic material and according to standard criteria. We feel that in these cases, radiology-guided fine needle aspiration may reduce this problem in some of these cases. Clinical correlation is strongly recommended in cases where suspicion of malignancy is high but cytologically reported as benign. For these cases, we recommend a repeat aspiration or radiologically-guided aspiration. There-fore, communication between the clinician and the cytopathologist is extremely important.</p>\r\n<p>\r\n	There were four false positive cases giving a rate of 6.6%. The reported false positive rates in the literature vary from 0.3% to 10% (Suresh et al 1995). This was not considered a major problem because these patients would have been operated based on other clinical parameters. Three goitres were cytologically diagnosed as papillary carcinoma of the thyroid. Review of these cases showed that these were due to interpretative errors. In these cases, diagnosis of papillary carcinoma of the thyroid was offered due to hypercellularity of the specimens together with scant amount of colloid and the occasional presence of longitudinal grooves accom-panied by intranuclear pseudoinclusion-like features. The other false positive case was a follicular adenoma misdiagnosed as papillary carcinoma of the thyroid. Because longitudinal grooves and intranuclear pseudoinclusions are occasionally ob-served in other thyroid lesions, Cibas (Cibas et al 2003) stressed the significance of pale, powdery chromatin pattern in papillary carcinoma (Figure 1) as a helpful distinguishing feature from the coarse chromatin pattern seen in follicular lesions (Figure 2). Awareness by the cyto-pathologists of this problem should reduce this particular diagnostic error in future.&nbsp;</p>\r\n<p>\r\n	Finally, even though we excluded the inconclusive cases from our calculations, it was interesting to note that we correctly assigned 36 (87.8%) out of 41 cases diagnosed as follicular neoplasms.&nbsp;&nbsp; Histo-logical diagnosis confirmed 31 cases of follicular adenoma and five cases of follicular carcinoma.&nbsp; Despite the extensive work done on separating follicular ade-noma and carcinoma on cytological basis, only marginal success has been achieved. Molecular technique is another potential area which may help resolve this problem in future. Molecules like CD44v6 and Galectin 3 seem to be promising markers to detect neoplastic thyroid epithelial cells (Matesa et al 2002, Inohara et al 1994, Ermac et al 1995). However, more work and results are needed before they can be routinely used clinically. One of the follicular neoplasms turned out to be a follicular variant of papillary carcinoma of the thyroid. This mistake is not common due to the presence of overlapping follicular and papillary features. Careful assessment of the nuclear cytological features is important to avoid future mistakes in diagnosis of this variant.</p>\r\n<p>\r\n	In conclusion, results of our study showed that our sensitivity, specificity and diagnostic accuracy were high, thus confirming the important role of&nbsp; fine needle aspiration cytology as&nbsp; the initial diagnostic utility in the management of thyroid nodules. Sampling error was the major cause of our false negativity, which may be reduced by incorporating the other clinical parameters as index of suspicion of malignancy and utilizing radiology-guided aspiration.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(389, 386, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n	<p>\r\n		In healthy life expectancy studies, women can expect to spend more years of their lives with functional limitation than men, both in developed and developing countries. With increasing gender difference in life expectancy, data at hand is insufficient to assess the extent of disability of older women in developing countries. In developed countries, studies showed that the prevalence of disability is less than 5% at 60-64 years, less than 10% at 70-74% and above 20% after 85 years. With more women reaching beyond 80</p>\r\n	<p>\r\n		years of life, there is concern on the quality of their extended lives in the 21<sup>st</sup> century [United Nations Statistic Division 2000].</p>\r\n	<p>\r\n		&nbsp;&nbsp; With the majority of women between 45 to 60 years being apparently well, it is opportune to counsel them at this juncture to undergo a general medical audit to detect and correct any underlying disease early. Currently in Malaysia, this subgroup (female) above 45 years comprised 10% of the population or about 1.25 million [Dept of Statistics 2002]. The perimenopause presented a challenge to the clinical practitioner as it offered opportunities for identification of risk factors for future diseases, indicators for the early presence of established disease with subsequent enhancement of life quality and care for women in the later years [Lobo 2004].</p>\r\n</div>\r\n<p>\r\n	&nbsp;&nbsp; Women between the ages of 40-59 have a 1 in 11 chance of developing some type of invasive cancers. In comparison, women from ages 1-39 have a 1 in 51 chance of being diagnosed with cancer [Givens and Ling 2003]. The most frequent cancers in Malaysian women were cancers of the breast, cervix, colon, ovary, leukaemias and lung. Cancers of ovary and corpus uteri, respectively made up 5% and 3.6% of all cancer cases [Looi et al 2004].</p>\r\n<p>\r\n	&nbsp;&nbsp; Endometrial cancer (EC) accounted for 3.8% of all cancers in women worldwide. EC is one of the commonest malignancies seen in 1-25% of women with unexpected postmenopausal bleeding, and caused fewer deaths compared to cancer of other reproductive organs. Almost 90% of women above 50 years old had abnormal bleeding as the commonest presenting symptom [Goldstein et al 2002; Parkin 1998]. Prior to 1982, diagnostic evaluation was routinely by diagnostic dilatation and curettage. Nowadays, transvaginal sono-graphy is becoming an increasingly popular tool for endometrial assessment with similar false positive rates as endometrial sampling in cancer detection. During ultra-sonography, the image of the endometrium showing thickened and heterogeneous areas was a predictive factor for endometrial malignancy [Gull et al 2003].</p>\r\n<p>\r\n	&nbsp;&nbsp; The overall five year survival rate was 70-80% in western countries, ranging from 90% for stage 1 patients to 20% for stage 4. The incidence rate rose steadily 5 to 10 years before menopause, peaked at 65-70 years and thereafter declined [Parkin 1998]. The menopausal transition or perimenopause was distinguished as a period of elevated serum estrogen and low levels of progesterone whereby a possible &ldquo;window of risks&rdquo; existed for development of EC. Studies have shown that prolonged inter-menstrual period, common in perimenopause lead to sustained E<sub>2</sub> and ir-regular progesterone levels [Hale et al 2002].</p>\r\n<p>\r\n	&nbsp;&nbsp; Ovarian cancer (OC) remained the deadliest of all gynaecological malign-nancies. In the United Kingdom, it accounted for nearly 6% of all deaths from cancerin females causing more deaths in women than for allother gynaecological malignancies combined.In United States, OC affected 1 in 56 women and caused 14,500 deaths annually. It was the fourth leading cause of cancer death in perimenopausal women, mainly due to the lack of symptoms in affected patients until the disease had spread. With the lack of early symptoms, 65-75% of cases presentedat an advanced stage of the disease [Lacey et al 2002; DePriest &amp; DeSimone 2003]. The overall five year relativesurvival rate was 28%, but the five year survival rate for stagesIII and IV combined was a dismal 10%. A five year survival rate greaterthan 90% may be achieved in a small minority whenthe disease was confined to the ovary at diagnosis [Austoker 1994].</p>\r\n<p>\r\n	&nbsp;&nbsp; Sonographic detection of abnormal postmenopausal ovarian volume or demonstration of deviation from the norm could assist the clinician in diagnosing early ovarian neoplasia. Currently, pelvic ultrasonography visualise and characterise adnexal masses with greater sensitivity and specificity than clinical palpation alone. Transvaginal sonography was recom-mended as the principal technique for final assessment of whether a subject should be referred for surgical investigations [DePriest et al 1997; Collins et al 1998].</p>\r\n<p>\r\n	&nbsp;&nbsp; To support the basis of selective screening, a study on disease free urban midlife women, non-users of HRT with intact uterus aged 45 years old and above was undertaken to provide normograms of ovarian volume, uterine size and endometrial thickness. The findings would hopefully provide health indices relevant to the local community.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Data for this study was obtained from entry assessment of subjects who participated in a lifestyle intervention program. Briefly, healthy women aged 45 years and above with intact uterus, non HRT users were recruited from November 1999 to October 2002 via flyers distributed to the residential areas around the hospital including subjects from other areas who responded after being informed by friends.</p>\r\n<p>\r\n	&nbsp;&nbsp; After an initial screening of 745 women by telephone, 522 subjects were accepted into the study. Exclusion criteria included pregnancy, immigrants, those on treatment for hypertension, diabetes mellitus, hyperlipidaemia and chronic illnesses. Those with secondary causes of osteoporosis such as a thyroid/endocrine condition e.g. Cushing&rsquo;s disease, hype-rthyroidism, hyperparathyroidism, mal-absorption, hypogonadism, and prolonged immobilisation were also excluded. Five women were later excluded for surgically induced menopause leaving only 517 subjects. The study protocol was approved by the hospital ethics committee as part of a Masters of O&amp;G study thesis [Nur Azurah 2003].</p>\r\n<p>\r\n	&nbsp;&nbsp; All subjects gave a written informed consent before participation in the study. Recruits were interviewed for medical history and examined by a designated physician. Height and weight were measured to the nearest 0.1 unit with a balance beam weighing machine and stadiometer [Health-O-Meter, USA]. Infor-mation acquired by questionnaires included personal characteristics and lifestyle. All activities were carried out in the general gynaecology clinic by trained personnel.</p>\r\n<p>\r\n	&nbsp;&nbsp; Pelvic ultrasound was performed transvaginally (TVS) by qualified sono-graphers using a 5.0 MHz transvaginal probe [Toshiba-Tosbe SSH140A, Tokyo, Japan]. Measurements of endometrium, uterus and ovaries were taken. Uterus and ovaries were measured along the major and minor axes in the transverse and longitudinal planes. Images and thickness of the endometrium were measured along with endometrial heterogenicity as a predictive factor for endometrial malign-nancy.</p>\r\n<p>\r\n	&nbsp;&nbsp; The prolate ellipsoid formula (width &times; height &times; thickness &times; 0.523) was used to calculate the ovarian volume (OV) and/or cyst. TVS results were deemed abnormal/ positive if premenopausal OV was &gt;20cm<sup>3</sup>; postmenopausal OV &gt;10cm<sup>3</sup> respectively and cyst volume &gt;10cm<sup>3</sup>. The presence of structures such as solid area or papillary projection extending into the cavity of a cystic ovarian tumour of any size; or any mixed (solid/cystic) component within a cystic ovarian tumour suggesting malign-nancy was noted.</p>\r\n<p>\r\n	&nbsp;&nbsp; Only unambiguous ultrasound measure-ments were taken into consideration. Data were analysed using SPSS for Windows version 11.0 software. Continuous varia-bles were expressed as mean and standard deviation, and compared by the Student&rsquo;s t test. Categorical data were compared using the Chi-squared test and relationships with Pearson correlation where appropriate. The differences were considered significant if p&lt;0.05.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Subject characteristics</em></p>\r\n<p>\r\n	The study sample comprised of 300 (58.0%) premenopaused and 217 (42.0%) postmenopaused women, similarly distributed between the major ethnic groups [Table 1]. Over two thirds were Chinese followed by Malays and Indians, reflecting an urban distribution as opposed to the general population whereby Malays were the major ethnic entity. The average age of participants was 51.27&plusmn;5.35 years old.</p>\r\n<p>\r\n	&nbsp;&nbsp; The mean and median age of menopause was 49.97&plusmn;2.90 years and 50.00 years respectively with no significant difference between the ethnic groups. The postmenopausal group, aged 55.53&plusmn;5.27 years was 7.4 years older than the premenopausal group, 48.14&plusmn;2.67 years (p&lt;0.0005). Malays were the youngest, followed by Chinese, Indians and others with respective mean age of 50.14&plusmn;4.33, 51.51&plusmn;5.53, 54.47&plusmn;7.83 and 57.00&plusmn;10.53, a significant difference (p=0.039).</p>\r\n<p>\r\n	&nbsp;&nbsp; The average and median age of menarche was 13.57&plusmn;1.73 and 13.00 years old. Indians reached menarche at 12.55&plusmn;1.62 years; earlier than Malays, Chinese and others with a respective mean of 13.36&plusmn;1.32, 13.72&plusmn;1.87 and 13.20&plusmn;0.84 years (p&lt;0.005). All other reproductive and demographic characteristics were not significantly different between ethnic groups or menopausal status.</p>\r\n<p>\r\n	&nbsp;&nbsp; The mean height, weight and the body mass index (BMI) of the sample population was 1.55&plusmn;0.05cm, 59.41&plusmn;9.50kg and 24.64&plusmn;3.89kg/m<sup>2</sup> respectively. The pre-menopausal group was similar in weight, height and BMI to the post-menopausal group. Malays were the heaviest, 62.39&plusmn;10.00kg with significantly higher BMI, 26.15&plusmn;4.30kg/m<sup>2</sup>, compared to the others (both p&lt;0.0005). Weight and BMI of Chinese, Indians and other races were 58.30&plusmn;9.14kg, 58.03&plusmn;7.75kg and 57.20&plusmn;9.76; 24.08&plusmn;3.60kg/m<sup>2</sup>, 23.98&plusmn;2.83 kg/m<sup>2</sup> and 23.61&plusmn;2.40kg/m<sup>2</sup> respectively. Overall, 56.3% had BMI between 18.5 to &lt;25 kg/m<sup>2</sup>, 32% were overweight and 8.6% obese.</p>\r\n<p>\r\n	<em>Uterine size</em></p>\r\n<p>\r\n	The average uterine size for the sample population was 7.21&plusmn;1.67 x 4.36&plusmn;1.30 cm. Premenopausal women had significantly larger uterus compared to postmeno-paused women (p&lt;0.0005). There was no significant difference in uterine measure-ments between the three major ethnic groups and atrophy of the uterus with menopause was demonstrated regardless of ethnicity [Table 2].</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Nearly a fifth (18.9%) of the sample population had fibromyoma, most being detected from ultrasonography as intramural and posterior. A quarter (25%) of premenopausal women had uterine fibroids compared to 10% post-menopaused which was statistically significant (p&lt;0.0005). There was no signi- ficant difference observed in the size of the fibroids and prevalence within the three ethnic groups [Table 3].</p>\r\n<p>\r\n	A normogram drawn of pre and postmenopausal women free of fibroids showed a gradual decrease in uterine measurements with age. Postmenopausal women demonstrated much smaller uterine measurements than premenopaused wo-men [Figure 1]. Length and width of the uterus was positively correlated to weight, height, BMI, education and income but negatively correlated to current age, age of menarche or being postmenopaused. The main predictors of uterine length (adjusted R<sup>2</sup>=0.380, p&lt;0.0005) from multiple regression analysis were current age (&szlig;=0.140, p&lt;0.009), menopausal status (&szlig;=0.496, p&lt;0.0005) and income (&szlig;=0.081, p&lt;0.043). Uterine width also showed similar associations.</p>\r\n<p>\r\n	&nbsp;&nbsp; The occurrence of fibroids was negatively correlated to current age, age of menarche, and postmenopausal state but the only significant predictor was meno-pausal state (&szlig;=0.189, p&lt;0.043; R<sup>2</sup>=0.033, p&lt;0.0005).</p>\r\n<p>\r\n	<em>Endometrial thickness (ET)</em></p>\r\n<p>\r\n	Mean ET for the sample was 6.36&plusmn;3.73mm [Table 4]. Premenopausal ET was significantly thicker than the post-menopaused group (p&lt; 0.0005). Indian women had a comparatively thicker ET than other ethnic groups but this is not statistically significant.</p>\r\n<p>\r\n	&nbsp;&nbsp; ET showed decreasing thickness with age in pre and postmenopausal women. Early postmenopausal women demon-strated ET of less than 5mm, eventually reaching 2mm in women of more than 70 years old. From these findings, a cut-off of less than 5.0mm was considered as clinically acceptable for normal post-menopausal ET regardless of ethnicity [Figure 2]. Utilising this cut off (less than 5mm) for postmenopausal women at 55 years old and above, 16.3% of post-menopausal women required further invest-tigation although none of the subjects showed endometrial cancer (EC) on endometrial sampling.</p>\r\n<p>\r\n	&nbsp;&nbsp; ET was positively correlated to weight, height, education and income but was negatively correlated to current age, age of menarche and being postmenopaused. Multiple linear regression analysis showed the significant predictors to be menopausal status (&szlig;=0.373, p&lt;0.0005) and current age (&szlig;=0.213, p&lt;0.0005) with adjusted R<sup>2</sup>=0.290 (p&lt;0.0005).</p>\r\n<p>\r\n	<em>Ovarian volume (OV)</em></p>\r\n<p>\r\n	The average right and left ovarian volume (OV) was 5.48&plusmn;7.85cm<sup>3</sup> and 5.80&plusmn;16.78cm<sup>3</sup>, being not statistically different[Table 5]. A significant difference in right ovarian volume (ROV) was observed between the three main ethnic groups (p&lt;0.005). Premenopausal Indian women had larger ROV compared to Malays and Chinese due to the presence of ovarian cyst with mean LOV being similar. In postmenopausal women, ROV and LOV were similar between the ethnic groups. Postmenopausal ROV was significantly smaller than the pre-menopaused (p&lt;0.0005) but LOV was not statistically different.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Ovarian cysts were seen in 12 women i.e. 2.9% of sample with a mean size of 65.14&plusmn;88.22cm<sup>3 </sup>[Table 6]. Cysts were slightly larger in postmenopausal women than in premenopaused although the premenopaused had a higher prevalence. The size and prevalence of asymptomatic ovarian tumour was not significantly different between premenopausal and postmenopausal women with no differences observed between the ethnic groups in this small number [Table 6].</p>\r\n<p>\r\n	A normogram plotted using mea-surement from women who were cyst-free, showed a gradual decline in pre-menopausal OV from about 7cm<sup>3</sup> at age 45-49 years to less than 4cm<sup>3 </sup>at age 55-59 years. ROV was comparatively larger in older premenopausal (&gt;50 years) com-pared to younger (&le;50 years) pre-menopausal women. Among post-menopausal women, OV declined from over 3cm<sup>3</sup> at age 50-54 to less than 1.5cm<sup>3</sup> at 70 years old. After 60 years of age, both ROV and LOV remained consistent at less than 3cm<sup>3</sup> [Figure 3].</p>\r\n<p>\r\n	&nbsp;&nbsp; Since there was no statistical difference in volume between the right and left ovary, correlation was reported for the largest ovary. OV was positively correlated to weight and negatively correlated to current age, age of menarche, days of menstrual cycle and being postmenopaused. The only predictor of ROV from multiple linear regression analysis was menopausal status (&szlig;=0.255, p&lt;0.0005) with adjusted R<sup>2</sup>=0.063 (p&lt;0.0005). Unlike ROV, the significant predictors for LOV were age of menarche (&szlig;=0.152, p&lt;0.004) and number of days in the menstrual cycle (&szlig;=0.107, p&lt;0.040) with adjusted R<sup>2</sup>=0.029 (p=0.002). Ovarian cyst volume showed positive correlation to age of menarche and age of 1<sup>st</sup> childbirth with the age of 1<sup>st</sup> childbirth (&szlig;=0.578, p=0.030) as the only predictor with adjusted R<sup>2</sup>=0.620 (p=0.014) in the final model.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; From this study, a clinically normal OV of &lt;7.5cm<sup>3</sup> and &lt;3.5cm<sup>3</sup> respectively for pre and postmenopausal Malaysian women aged 45 and above was unquestionably demonstrated. Ovarian tumour was suspected when ovaries were larger in size than the clinical norm by two standard deviations i.e. over 20cm<sup>3</sup> for pre-menopause and10cm<sup>3</sup> for post-menopaused women. Utilising this cut-off index, 3.0% and 3.4% premenopausal and postmenopausal women respectively would require follow ups. Older postmenopausal women aged 55 years old and above showed a higher incidence of deviation from norm, i.e. 4.5%. Any enlargement in the ovaries in the postmenopause was considered ominous requiring vigilant monitoring. Follow ups showed that except for a larger volume, no subject had the presence of structures associated with malignancies.</p>\r\n<div>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		<em>Uterine size and uterine leiomyomata </em></p>\r\n	<p>\r\n		Uterine size of women in this study was comparable to other reported studies [Merz et al 1996]. The size of uterus was influenced by the presence of uterine fibroids which prevailed in 25 to 77% women of various populations [Stewart 2001]. In a study of 95,061 premenopausal nurses, the age-standardised rates of ultrasound or hysterectomy confirmed diagnoses of uterine fibroids per 1000 women years were 8.9 among white women, 30.6 among black women, 11.0 among Hispanics and 8.0 among Asians with peak incidence ranging from 40-44 years [Marshall et al 1997]. In comparison, 19% of all women in this study had asymptomatic fibroids, more common in</p>\r\n	<p>\r\n		premenopaused (25%) compared to the postmenopaused (10%).&nbsp; This was higher than the 4.9% prevalence reported for 164 women who attended an urban menopause clinic in Malaysia [Damodaran et al 2000]. In comparison, the reported prevalence in Thai women was 45% in the premenopaused and 6.3% in post-menopaused [Jirapinyo et al 1998].</p>\r\n	<p>\r\n		&nbsp;&nbsp; Despite the prevalence, very little is known about this condition as few studies had been carried out probably due to its benign nature with few clearly established risk factors for fibroids [Baird 2004]. A significant positive association was seen between its incidence with age, race, body mass index, years since last term birth, history of infertility and current alcohol consumption. Significant inverse asso-ciation was seen between age at menarche, age at first term birth, age at first oral contraceptive use and never being married [Marshall et al 1997]. In con-cordance, this study found that fibroids were negatively correlated to current age, age of menarche, and postmenopausal<br clear="all" />\r\n		state with the only significant predictor being menopause.</p>\r\n	<p>\r\n		<strong>Endometrial cancer (EC)</strong></p>\r\n	<p>\r\n		As this study recruited only disease-free, non-users of HRT with intact uterus, none presented with EC but postmenopausal endometrium thickness cut-off of 5mm was proposed based on the normogram, similar to the consensus made by the Society of Radiologists in Ultrasound Consensus Conference Statement on Postmenopausal bleeding, the recommended cut-off point to rule out malignancy in symptomatic postmenopausal women [Goldstein et al 2002] as thin endometrium was found almost never to harbour carcinoma [Gull et al 2003].</p>\r\n	<p>\r\n		&nbsp;&nbsp; In a meta-analysis, when post-meno-pausal women with EC presented with vaginal bleeding, the median ET was found to be 3.7 times thicker than those unaffected. The detection rate was 96% with a 50% false positive rate which in the authors&rsquo; opinion, still required invasive diagnostic testing in order not to miss 4% of EC [Tabor et al 2002]. Not much was known about EC in Malaysia except it being among the ten commonest malign-nancies [Lim 2002].</p>\r\n	<p>\r\n		&nbsp;&nbsp; Known factors that affected the endometrium were obesity, reproductive characteristics, certain medical conditions and cigarette smoking which may be explained partly by their effect on estrogen and progesterone [Hale et al 2002]. In this study, ET was positively correlated to weight, height, education and income. ET was negatively correlated to current age, age of menarche and being post-meno-paused. Multiple linear regression analysis showed the significant predictors to be menopausal status and current age. The relationship between ET and variables such as obesity and smoking was not observed as most of our subjects did not smoke (&gt;97%) and few were grossly overweight.</p>\r\n	<p>\r\n		<strong>Ovarian cancer (OC)</strong></p>\r\n	<p>\r\n		Results from this study showed a decline of OV with age in premenopausal and postmenopausal women not receiving HRT. The sizes of ovaries concurred with those from other studies [Opperman et al 2003; Pavlik et al 2000] and consequently similar clinical cut-off values were considered applicable to our population. Zalel et al [1996] showed an increased OV of more than two standard deviations from norm for each postmenopausal age were highly suggestive of malignancy. This deviation from norm was striking in malignant tumour (100%) compared to benign (86%) with proposed upper limit for OV in premenopaused women as 20cm<sup>3</sup> and for postmenopaused as 10cm<sup>3</sup> [Merz et al 1996]. In expert hands, OV and morphologic evaluation of complexity was shown to be inexpensive and accurate for differentiating benign from malignant tumours with sensitivity, specificity, PPV and NPV of 0.981, 0.808, 0.409 and 0.997 respectively [Ueland et al 2003].</p>\r\n	<p>\r\n		&nbsp;&nbsp; In this sample we did not encounter any patient with ovarian malignancy and hence could not provide a practical model. Ovarian cysts were seen in 2.9% of our subjects, slightly more prevalent among premenopausal women. Studies had shown that persistent lesions could be detected in about 3.6% of self referred, asymptomatic women at the first attendance of ultrasound screening. About 1.2% of screened subjects would have one benign tumour. The prevalence of OC was about 0.004%, more prevalent among postmenopausal women (over 94% of cases occurred in women over 40 years old) [Collins et al 1998]. In our experience, 69% of all ovarian tumours were benign with a younger peak age in the 3rd decade and most cancers were detected in the 4th to 5th decade. Tumours were rare before the 2nd decade and 77% were in the reproductive age [Thanikasalam et al 1992].</p>\r\n	<p>\r\n		&nbsp;&nbsp; OV was shown to be associated with age, menopausal status, phases of the menstrual cycle and hormonal milieu during menopausal transition [Merz et al 1996; Opperman et al 2003; Giacobbe et al 2004], variables similarly found in this study. Possibly due to the higher presence of cyst on the right ovary, the predictors of ROV differed from LOV being only menopausal status whereas significant predictors of LOV were age of menarche and days of the menstrual cycle.</p>\r\n	<p>\r\n		&nbsp;&nbsp; Data from this study did not reflect the general Malaysian population as the sample consisted of urban women with a Chinese majority instead of the usual population ratio. The sampled population had higher income, better education and access to health care. Nevertheless, this obtained data would be useful basis for further investigations particularly in high risk groups. Obtaining a complete data base is essential for delivering appropriate preventive services as incomplete data may result in inaccurate risk assessment resulting in inappropriate interventions.</p>\r\n	<p>\r\n		&nbsp;&nbsp; It is known that women with a first degree family history of breast cancer (BC) had a 1.7 fold increased risk of OC; an association with reciprocal ramifications.&nbsp; An increased risk of 2.2 fold was seen in women with a first degree relative affected with OC. Corresponding risks of 7.1 to 10.7 fold were observed for those with two or three affected relatives. Screening a population for OC with at least one first degree affected relative gave a prevalence of 3.8/1000 women. Screening women with BC/OC diseases gave a prevalence of 50/1000 [Collins et al 1998]. Thus OV and ET normogram should be made available for the local population to enable clinicians to tailor subsequent management and counselling.</p>\r\n	<p>\r\n		&nbsp;&nbsp; In summary, the findings showed that the uterus atrophy with age and menopause regardless of ethnicity. Nearly one fifth of all subjects had uterine fibroids, significantly more among premenopausal women compared to postmenopaused (25% vs 10%). Early postmenopausal women demonstrated ET of &lt;5mm, eventually reaching 2mm in those &gt;70 years old. A cut-off &lt;5.0mm was considered clinically acceptable as normal postmenopausal ET regardless of ethnicity. With a gradual decline in OV with age and menopause from about 7cm<sup>3</sup> at age 45-49 years to less than 1.5cm<sup>3</sup> at 70 years old, a clinical normal OV of &lt;7.5 cm<sup>3</sup> and &lt;3.5cm<sup>3</sup> respectively was suggested for pre and postmenopausal Malaysian women. Benign ovarian cysts were found in 2.9%.</p>\r\n	<p>\r\n		<strong>ACKNOWLEDGEMENT</strong></p>\r\n	<p>\r\n		The authors gratefully acknowledge and thank the following for their assistance &ndash; Prof MA Jamil Yassin, Anson Ismail (Dept O&amp;G, Ultrasound Unit); Ng Choo Bing, Ooi Teng Hong, Shuhaila Shohaimi, Hairun Bee (Dept of Rehabilitation); Prof Nor Azmi Kamaruddin, Norlinda Daut (Dept of Medicine, Bonescan Lab); Prof Nafikudin Hj Mahmud, Siti Zakiah Othman (Dept of Radiology, Radiology Unit), Prof Md Idris Mohd Nor (Medical Statistics, Dept of Community Health), Ruth Teh and Pon Lai Wan (data collection). Funding for the project came from USM Top Down Program IRPA No:06-02-05-9002 for which we would like to thank the program directors, Dato&rsquo; Prof Mafauzy Mohamed and Dato&rsquo; Prof Mustafa Embong.</p>\r\n</div>\r\n', 1),
(390, 387, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Smoking is a major public health problem in the developing world. Rapid socio-economic development in developing countries is accompanied by consistent increase in tobacco consumption (WHO 1996).&nbsp; In the 1990s, a world total of three million people died each year from tobacco induced diseases (Anthony et al. 1997).</p>\r\n<p>\r\n	Over 75% of adult smokers reported that their first smoking experience began during adolescence (Anthony et al. 1994).&nbsp; Smoking is a great concern among adolescents as it is related to many chronic diseases and mortality which become evident only after two or three decades of tobacco use. It is also associated with other risky behaviours such as other drug abuse, truancy and physical fighting (Epps et al. 1995, Kulig et al. 2005). Once a regular smoking pattern is established, it is difficult to cease, with onset during adolescence being highly predictive of lifetime use (Heischober et al. 1997).</p>\r\n<p>\r\n	There are not many studies done in Malaysia previously which&nbsp;&nbsp; looked into the reasons for smoking and the contributing factors influencing it. Thus, the objective of this study was to determine the prevalence of smoking among upper secondary schoolboys, reasons for smoking and the contributing factors to the commonest reason given. The findings in this study could be used to improve strategies in smoking prevention programmes for adolescent schoolboys.</p>\r\n<p>\r\n	<strong>SUBJECTS AND METHODS</strong></p>\r\n<p>\r\n	This was a cross-sectional study conducted among upper secondary school-boys (Form 4 and Form 5 students). Three secondary schools in the District of Hulu Langat, Selangor were selected randomly. From each school, an equal number of students was chosen randomly from the class registration lists. Form 4 and form 5 students from all ethnicities were included in the study. Consent was obtained from the students and their parents. Those who refused to give consent to participate in the study were excluded. The students were then given a self-administered question-naire. The questionnaire was pre-tested and consisted of questions regarding socio-demographic characteristics, age at which smoking was initiated, smoking among family members as well as reasons for initiating and continuing smoking. Non-smokers were those who never smoke for at least one month while smokers are those who smoked at least one cigarette for the preceding one month (Khadijah et al. 2000). Data entry and analysis was done using Epi-info 2002. A confidence interval of 95% and p value of less than 0.05 were considered as significant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 343 students were included in the study. The respondents comprised 57% Malays, 36% Chinese and 6.7% Indians. Their ages ranged&nbsp;&nbsp; between 16 and 18 years old; 57% of them were aged sixteen years, 40% seventeen years and 3% eighteen years. The majority of them were from families with a family income ranging from RM500 to RM2000 (66%).</p>\r\n<p>\r\n	The prevalence of smoking among the boys was 37%. Although the majority of them initiated smoking at 13 to 15 years old (66%), 21% of them began smoking during primary school (<u>&lt;</u>12 years old) (Table 1). Among the smokers, the majority of them smoked more than 10 cigarettes per day (57%) (Table 1). There was a significant association between adolescent smoking and smoking among family members (p&lt;0.05).</p>\r\n<p>\r\n	The common reasons given by the respondents for initiating smoking and continuing smoking varied widely. The most frequent reasons for initiating smoking were curiosity (69.3%) and peer pressure (51%) while stress (70%) was reported as the commonest reason for continuing smoking followed by addiction (49%) (Table 2). The students reported schoolwork as the most important factor which contributed to their stress (Table 2).</p>\r\n<p>\r\n	The majority of cigarette smokers initiate smoking during their adolescence (Anthony et al. 1994, Heischober 1997). The prevalence of smoking among adolescent schoolboys in Asian countries ranges from 3% to 30% in the 1980s (Thambipillay 1985, Emmanuel et al.1990, Stone et al. 1992, Habibul et al. 1998). The National Health Morbidity Survey done in Malaysia reported an increasing trend in the prevalence of smoking among adults from 39% in 1986 (MOH Malaysia 1986) to 49% in 1996 (MOH Malaysia 1996).&nbsp; However, data on the prevalence of smoking among adolescents was not available. In 1997, a study done in Kota Bharu, Kelantan reported the prevalence of cigarette smoking among male secondary students as 33.2% (Khadijah et al. 2000).&nbsp;In the present study, the prevalence of smoking among male upper secondary school students was 37%.&nbsp;&nbsp; Although the setting in the study by Khadijah et al (2000) was different, there may be a rising trend in cigarette smoking among adolescent boys; thus it is essential for health care providers to address this issue and improve the preventive intervention strategies against smoking.&nbsp;</p>\r\n<p>\r\n	This study showed that 21% of the adolescent boys who smoked initiated smoking at 12 years of age or younger (during primary school). This indicates that education regarding smoking, emphasizing on its negative consequences, should begin early in the primary schools. It should be part of the primary school curriculum and students should be taught ways to say &ldquo;no&rdquo; to tobacco smoking. Many studies have reported that those who started smoking early in life have greater difficulty in stopping. (Epps et al. 1995, Heischober et al. 1997, Bruvold et al 1993, Siquera 2000). They are also more likely to become heavy smokers and are at higher risk of developing smoking-related diseases than those who begin at a later age (MOH Malaysia 1996, Anda et al. 1999). The children who started smoking at an earlier age may be due to earlier exposure to tobacco environment either at home, school or in the community. Electronic media, for example television and movies, also have great influence on children. It is the responsibility of parents and other adults to supervise and educate children that smoking is an unfavourable habit which should not be attempted.</p>\r\n<p>\r\n	Smoking habits among adolescents had&nbsp;&nbsp; been shown to be influenced by parents and other adult role models who smoke (Heischober et al. 1997, Bruvold et al. 1997). This study also showed a significant association between adolescent smoking and smoking among family members. Thus, health practitioners should increase awareness among parents that they are important role models for their children and should ensure that they do not influence their children towards smoking.&nbsp;</p>\r\n<p>\r\n	In this study, the most frequent reasons given by the adolescents for initiating smoking were curiosity (69%) and peer pressure (51%). Many adolescents begin smoking to fit into the society or peer group (Heischober et al. 1997, MOH Malaysia 1996). Adolescents tend to experiment smoking together and by doing so they believe that they are more accepted by their peers (Reininger et al. 2005). It is important that health professionals understand the psychosocial develop-mental process in adolescence. During adolescence, there is lack of impulse control and there is an increase desire to experiment new experiences; thus the inclination to risk-taking behaviours such as smoking. This explains the commonest reason reported by the respondents for initiating smoking which was curiosity. In addition, during the middle adolescence stage, the role of peer groups becomes more evident and they are susceptible to peer pressure. Adolescents tend to be intensely involved in their peer subculture by conforming with peer values and codes (Reininger et al. 2005). They should learn to be assertive to say &lsquo;no&rsquo; to smoking and influence peers against smoking. Other common reasons given by the adolescents for initiating smoking were stress (42%) and &lsquo;feel more mature&rsquo; (25%). Adolescents should be educated that smoking cigarettes is not the solution to alleviate stress or solve their problems but it can lead to many other negative con-sequences. &lsquo;Feel more mature&rsquo; is an expected response by adolescents as they are in the process of forming an identity. They are frequently exposed to smoking adults and subconsciously identify smoking with maturity which is a misperception. Thus it is important to expose adolescents to better adult role-models and a healthier community.</p>\r\n<p>\r\n	The commonest reason given by the adolescents in this study for continuing smoking was stress (70%). The main contributing factors to stress reported by them were schoolwork and peers. Demands from schoolwork such as heavy homework assignments, difficulty in understanding lessons, getting poor grades and high parental expectations contribute to the stress in many adolescents. In addition, peers play an important role in&nbsp;&nbsp; adolescence. Misunderstanding among friends, peer pressure and the need to be accepted by friends are stressors to adolescents. Stress can eventually lead to anxiety or depression in extreme situations. Hence, it is essential that adolescents learn how to deal with stress effectively instead of ignoring it or indulging in bad habits such as cigarette smoking or other substance abuse.</p>\r\n<p>\r\n	The significance of good coping skills among adolescents is of utmost importance in the planning for anti-smoking campaigns. Current strategies to prevent adolescents from engaging in high risk behaviours include a focus on building protective factors such as strong family relationships, religious beliefs, parental monitoring, adult role models and engage-ment in structured activities (Reininger et al. 2005). Effective treatment strategies to cease smoking must take place within the context of the youth&rsquo;s normal daily life and resist all the factors that promote continuance (Bruvold et al. 1993). Hwang (2005) examined 65 adolescent psycho-social smoking prevention programmes. They reported that knowledge had the highest short-term effect (less than a year) but rapidly decreased in the long term. Behavioural effect was the most meaningful prevention programme as it persists over a three year period. They also reported that smoking reduction rates were increased by using either cognitive behaviour or life skills programme modali-ties in a school-community-incorporated programme setting (Hwang et al.&nbsp;&nbsp; 2005).</p>\r\n<p>\r\n	There are limitations in this study.&nbsp; This study included only three secondary schools in a single area. Hence the findings in this study cannot be generalized to represent the country.&nbsp; There may be recall bias among the adolescents while answering the questionnaire and some may not have revealed the true facts of their smoking behaviour.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Early smoking intervention strategies are necessary as younger boys are now involved in smoking.&nbsp; The initiatives should begin early in primary school to reduce the prevalence of smoking among adolescents.&nbsp; Educating the adolescents on being assertive and acquiring effective coping strategies in managing stress are important issues to be included in the preventive strategies. Health care providers should take every opportunity to screen smoking among their adolescent patients. They should incorporate cigarette smoking prevention into daily practice, acquire the necessary skills to identify young people at risk of smoking and provide assessment, intervention and treatment when necessary.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(391, 388, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Exposure to infectious blood and hazardous fluids is one of the most reported events at Hospital University Kebangsaan Malaysia (HUKM). An average of two accidental sharps injuries among health care workers have been reported every month (infection control manual 1998). A great deal of anxiety (often out of proportion) can arise and at the same time the rights of the patient who was the source of infection should not be compromised.</p>\r\n<p>\r\n	&nbsp;&nbsp; Sharps injury may cause a number of potentially fatal infections with blood &ndash; borne pathogens. Stringer et al, (2001) have stated that when infected by needles, the risk can be high depending on the quantity of virus present in the blood of the source person at the time of the injury, the depth of the injury and the volume of body fluids breaching the cutaneous barrier. Stringer et al, (2001) further mentioned that donning of gloves may reduce the amount of solution introduced into the wound because the needle may lose half its volume on the glove surface.</p>\r\n<p>\r\n	&nbsp;&nbsp; Occupational exposures should be considered urgent medical events to ensure timely administration of post exposure prophylaxis. Unfortunately some of the health care workers in HUKM do not report their injuries (Infection Control Unit,HUKM). It is either because they do not know where to report or are just ignorant to the dangers of the injury.</p>\r\n<p>\r\n	&nbsp;&nbsp; The other reasons for not reporting could be due to the fear of disciplinary action due to the negligence on the part of the health care workers, doubts about their profession on whether they will be allowed to continue giving care to patients if they were confirmed to have the disease, and most of all, this may be a potential stigma attached with the fear and denial of contracting a serious disease from a contagious person. Nurses may also not report the injury immediately if the ward is rather busy especially during night shift as they may not have the time to leave the ward.</p>\r\n<p>\r\n	&nbsp;&nbsp; According to Jantan (2000) most of the sharps injuries were due to the absence of sharps bin at the site of the procedure and neglected needles are left in trays, kidney dishes, among drapes and among trash. It also stated that non compliance or failure to adhere to guidelines can become a contributory factor to needle stick injuries.</p>\r\n<p>\r\n	&nbsp;&nbsp; Davies (2000) stated that in the operating theatre, 39% of the injuries were self inflicted while 61% were inflicted by the surgeon or assistant and the majority of the injuries occurred during transfer of sharps between personnel for eg. direct hand transfer of needles or scalpel blades on handle.</p>\r\n<p>\r\n	&nbsp;&nbsp; The cleaners were injured while clearing sharps bins or while cleaning bed linens where cannulas may have accidentally dropped out of patients&rsquo; hands or care-lessly left behind by an attending doctor or nurse.</p>\r\n<p>\r\n	The sharps are sometimes disposed into ordinary disposal bags which are sometimes used for general purposes instead of disposing them into prepared yellow sharps bins. Disposing of sharps into any container other than the sharps bin is a very irresponsible act which can result in serious injuries to other persons. Such an act can be considered as negligence in carrying out one&rsquo;s duties. Syringes and sharps may not have been disposed of appropriately during emergency situation and all health care workers need to be vigilant clearing sharps after such events.</p>\r\n<p>\r\n	&nbsp;&nbsp; The ongoing stressor caused by the sharps injuries either due to negligence on the part of the victims or those working with them reinforces the need for continued administrative support through&nbsp; provision of protective equipment to limit occupational exposure and encouraging prevention and treatment strategies in the face of occupational exposure.</p>\r\n<p>\r\n	&nbsp;&nbsp; The study was carried out with the hope that it will contribute to the better management of the health care workers who are exposed to sharps injury and should assist in furthering the debate on how best to prevent such injuries. It is hoped that by identifying the problems encountered by the injured staff, appro-priate treatment can be given to prevent further anxiety, stress and incapacitation.</p>\r\n<p>\r\n	&nbsp;&nbsp; The present study was carried out to:</p>\r\n<p style="margin-left:.5in;">\r\n	a.&nbsp;&nbsp;&nbsp;&nbsp; determine the prevalence of sharps injury in HUKM.</p>\r\n<p style="margin-left:.5in;">\r\n	b.&nbsp;&nbsp;&nbsp; describe some of the emotional problems and experiences by staff who had encountered sharps injury.</p>\r\n<p style="margin-left:.5in;">\r\n	c.&nbsp;&nbsp;&nbsp;&nbsp; identify contributing factors to the occurrence of the sharps injury and to suggest ways on which the injuries can be prevented.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	The study was conducted using an exploratory, descriptive design with data collected via a semi-structured interview and a self prepared questionnaire which had been tested for validity and reliability. The study was conducted in several wards and the general operating theatres in HUKM. Approval to conduct the study was obtained from the Medical Research Ethics Committee and the director of HUKM.</p>\r\n<p>\r\n	&nbsp;&nbsp; The researcher used an exploratory/ descriptive approach because this method seeks to explore, describe and expand knowledge about the experiences of the respondents. The questionnaire used for collecting data was known as the Sharps injury Experience Questionnaire (SIEQ). This is a 28 item questionnaire with semi-structured and open-ended questions. The questionnaire had three parts: Part one was on the demographic data of the respondents which consisted of age, nationality, place of work, occupational and marital status.&nbsp; The second section was on respondents experience after a sharps injury. The third section of the questionnaire tested the feelings of the respondents after the sharps injury.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 64 respondents participated in the study despite its sensitive nature. The majority of the respondents were within the age group of 22 to 26 years (n=35 or 54.7%) and seven were above 40 years of age. Malay staff comprised the majority of the respondents (n=47 or 73.4%) and the staff nurses comprised 51.6% of the sample (Table 1).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp; Forty-four of the respondents incurred injury only once, 14 were injured twice and 6 of them were injured more than twice and out of the 9 doctors who participated in the study, 6 were injured once and 3 twice (Table 2). Several reasons have been identified as the cause of the sharps injury (Table 3).</p>\r\n<p>\r\n	&nbsp;&nbsp; Neglected needles in the kidney dish caused the majority of the injuries followed by recapping of needles. Handling garbage bags was one of the hazards posed to the janitors.&nbsp; The respondents were asked to identify their status after the sharps injury from a set of prepared answers. For this item, most of the respondents gave multiple answers. The results however showed that 43 of the respondents were anxious that something awful might happen and 9 felt like quitting their job.&nbsp; Some suggestions were given by the respondents to reduce the sharps injury in HUKM (Table 5).</p>\r\n<p>\r\n	&nbsp;&nbsp; Creating awareness among the staff through seminars, courses and posters were considered to be the most important among the respondents (n=29) and 15 of them suggested that proper and sufficient devices should be introduced to prevent injury by sharps.</p>\r\n<p>\r\n	<strong>DISCUSSION AND CONCLUSION</strong></p>\r\n<p>\r\n	The results of the present study suggest that the most frequently injured employees of HUKM were the staff nurses followed by the nursing students; often they were injured more than twice. Hanrahan et al, (1997) reported that two-thirds of all reported injuries occurred in nurses, they were involved with patients more often than any other hospital staff and sometimes they did not take any precautionary measures when performing hazardous procedures because they did not know the status of the patient.</p>\r\n<p>\r\n	&nbsp;&nbsp; It is basically believed that a small needle prick injury is harmless but stringer et al, (2001) stated that in a small needle prick injury a small amount of blood from the sharps can be introduced into the puncture site. This may not cause an infection to the health care worker but can pose a danger to the health care worker depending on the quantity of virus present in the blood of the source person at the time of the injury. The authors also found that most of the staff nurses were injured due to negligence of health care staff who left used sharps on trays or kidney dishes among trash like paper and gauze which covered the sharps. This is consistent with the findings of Wang et al, (2000), who found that the health care worker suffered injuries in the process of sorting and cleaning the instruments after use.</p>\r\n<p>\r\n	&nbsp;&nbsp; Injuries did not occur at random, nor were the substantial rates of needle stick injuries caused by a few health care workers who were injured repeatedly. Certain nursing practices were related to the likelihood of being injured and in this study, recapping needles appeared to be one of the most risky practices. Recapping persisted in spite of well documented hazards and prevention recommendations against the practice. Thurn&rsquo;s (1997) study supports this finding where recapping and improper disposal of sharps accounted for one-third of all work related injuries.</p>\r\n<p>\r\n	&nbsp;&nbsp; One doctor blamed tiredness and hunger for the sharps injury. She too mentioned that sometimes the doctors had too many wards to cover and post-call doctors did not get a day off the next day despite being busy the previous day. Osborn et al., (1999) believed that fatigue and stress impeded the practice of safe procedures. Hence greater enforcement of scheduled day off and adequate opportunity for sleep seemed important.</p>\r\n<p>\r\n	&nbsp;&nbsp; Disposal of used sharps into sharps bin also poses a danger because sometimes the sharps bins overflow, especially over the weekends. This is consistent with the study carried out&nbsp; by Gershon et al., (2000) where three health care workers were injured while trying to dispose contaminated needles because they failed to see&nbsp; another needle sticking out of the container. The sharp bins should only be &lsquo;three-quarter&rsquo; filled (OSHA) guidelines.</p>\r\n<p>\r\n	&nbsp;&nbsp; Throwing sharp items into garbage bags should be avoided and this should be instilled in the minds of all the health care workers. The present study shows that most of the cleaners got pricked by needles disposed into garbage bags. The janitors in the process of squashing the bags got pricked.</p>\r\n<p>\r\n	&nbsp;&nbsp; Health care workers also get injured in the process of blindly passing instruments. This was especially common in the operating theatre (n=3) and accords with the study carried out by Johnson (1996) who stated that emergency situations and careless passing of instruments are a frequent cause of injuries among health care workers.</p>\r\n<p>\r\n	&nbsp;&nbsp; Harnage (2000) stated that even if no infection is transmitted, the emotional impact of having been pricked by a contaminated sharp can be daunting and the uncertainties can lead to long term anxiety and may require further counselling. In the present study although 90% of the respondents were upset about the incident, only one of them was sent for counselling while the others were consoled by friends and colleagues.</p>\r\n<p>\r\n	&nbsp;&nbsp; The researcher is of the opinion that counselling might give peace of mind to some of the health care workers which in turn will give them the confidence to carry on with their duties. All and Sullivan (1997) had stated in their study that the fear of acquiring a blood borne disease generated anxiety to the extent that the nurses could not show empathy and compassion to the patients. The fear of contracting the disease would prevent the health care workers from giving maximum care to the patients which in turn could affect the health care system quite badly.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; In conclusion, it can be said that the present study provides evidence that the risk of sharps injury among health care workers is related to working conditions and to a certain extent carelessness on the part of the staff themselves who are handling the sharps. Although staff shortage in the hospital are more important issues, the consideration of organizational factors as well as equipment and other procedural aspects of care delivery will yield the best approaches in sharps injury protection and prevent further traumatic depression among certain injured health care workers. It can be concluded that sharps injury will always be a problem and will continue to increase the risk of serious and fatal diseases among health care workers if habits do not change. Greater efforts are needed by the administration to prevent such exposures to reduce work place hazards. The health care system must be made as safe as possible for both patients and health care workers. A new policy on work place injury prevention may achieve a high level of health care provider safety.</p>\r\n', 1),
(392, 389, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	In this high technology and modem era, where high performance is expected to be the norm, many organizations demand a high level of quality, service and overall business success. Therefore, the pressure is felt on individuals at all levels of organization. Performance targets are becoming tougher to meet with each succeeding year and the management staff is experiencing difficulty in fulfilling the challenge. Work stress is common among staff and there are grave concerns on how it may affect health.</p>\r\n<p>\r\n	&nbsp;&nbsp; The demand and pressure in the workplace place is an important source of stress. The factors in the workplace that have been to be associated with stress and health risks can be categorized into those&nbsp; related to the content of work and those&nbsp; related to social and organizational context of work (Michie, 2002). The factors that are intrinsic to the job include long hours, work overload, time pressure, difficult or complex tasks, lack of breaks, lack of variety, and poor working environment.</p>\r\n<p>\r\n	&nbsp;&nbsp; Ivancevich and Matteson (1980) suggested that the increase in mental health problems reported by workers in industrialized countries was a result of psychological stress and excessive job demands in the workplace. Karasek (1980) reported that job stress could be divided into two factors, job demand and job decision latitude or job control. He explained that a high job demand and a low decision latitude state were the most stressful. Much of the research regarding work stress was based on the model developed and refined by Karasek. He proposed that work-related mental strain and the associated psychiatric disorder may result from combinations of, and interactions between, four different employment factors: heavy job demands, limited input to decision making processes, lack of skill discretion within the job and poor work-based social support (Mausner-Dorsch &amp; Eaton, 2000). These factors, in particular those concerning decision making and lack of social support have been found to be most challenging for those in the lower grades of employment and to be less common among employees in higher ranking positions (Cropley et al, 1999).</p>\r\n<p>\r\n	&nbsp;&nbsp; Persistent stress had been linked to many physiological problems. Initially, the effects may be psychosomatic, but with continued stress, the symptoms can present as actual organic disease for example gastric or duodenal ulcers and coronary heart disease. Fraser (1997) grouped human reactions to workplace stress into 5 categories:&nbsp; (i) subjective or emotional, for example anxiety and aggression; (ii) behavioral, for example trembling sleep problems; (iii) cognitive, for example lack of concentration and inability to make decisions; (iv) physiological, for example increased heart rate and blood pressure: (v) organizational, for example absenteeism and poor productivity.</p>\r\n<p>\r\n	&nbsp;&nbsp; The international tobacco company in the study is one of the largest tobacco companies in Malaysia. At the time of study, this company had 1,200 employees -340 management employees and 860 employees who are involved in the full spectrum of the tobacco industry, from leaf buying, processing and manufacturing, marketing and distribution. There are nine management departments in the company, i.e. human resources and security affairs, finance, trade marketing, brand marketing, information technology, production, supply chain, corporate and regulatory affairs, legal and leaf departments.</p>\r\n<p>\r\n	&nbsp;&nbsp; The objectives of the study were to examine the prevalence of work stress among the management staff in the international tobacco company and to investigate the factors contributing to work stress. The justification of the study was to find out the prevalence of stress because of the stressful nature of work, and secondly, no similar study has ever been done in a tobacco company in Malaysia before.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross-sectional study was conducted in May 2004. Self-administered question-naires were used in the study, involving 185 management staff. The inclusion criteria were all permanent management executives and managers. The exclusion criteria were directors, expatriates, contract staff, and temporary staff. The sampling method was universal sampling. The subjects were given a questionnaire based on the Personal Stress Inventory by O&rsquo;Donell (1984). The questionnaire com-prised of 4 parts. The first part consisted of socio-demographic data and job history. Socio-demographic measurements in-cluded gender, age, marital status, number of children, medical illness and smoking habits. The job data included occupation, department, salary, length of service and job promotion. The second part of the questionnaire measured stress level based on reported symptoms.</p>\r\n<p>\r\n	It comprised of 52 items, which are the symptoms of stress. The third part con-sisted of 12 items which were questions on the perception of personal stressors.&nbsp; The last part of the questionnaire were&nbsp; questions on&nbsp; stressors at workplace relating organizational policy, organi-zational structure, organizational process, work environment, recognition and appreciation and work process. It also included two questions that gauge stress at work which require the respondents to write down their work stress factors and their personal methods for relieving stress.</p>\r\n<p>\r\n	<strong>Statistical analysis</strong></p>\r\n<p>\r\n	Data were analyzed with SPSS version 11.5. The significant level was set at 0.05.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	From 185 questionnaires distributed, 150 questionnaires were completed and returned, giving a response rate of 81%. In this study, 78 (52%) respondents were managers and 72 (48%) were executives. The age group of 31-40 years old consisted 44 (29%) of respondents while 16 respondents (10.7%) were in age group of 21-25 years. There were 99 (66%) male and 51 (34%) female respondents in the study. A total of 93 (62%) of the respondents were married and 55 (36.7%) were single, and 2(1.3%) were divorced or separated. The demographic data for the subjects in the survey are shown in Table 1.</p>\r\n<p>\r\n	The prevalence of stress among managers was 67.9% and executives were 68.1%. There was no significant difference in the level of stress between the executives and the managers in the company. There was a significant relationship between stress and salary, number of children (p&lt; 0.05).</p>\r\n<p>\r\n	The stressors at workplace (working environment, work process, organizational structure, organizational policy, organiza-tional process and acknowledgement) have statistical significant relationship with stress. Table 3 lists the mean stress score for work stressors. A bivariate correlation was done to test the correlation between perceptions of stress at work with stress symptoms and felt like resign. The results showed that there was significant correlation (p&lt;0.05).</p>\r\n<p>\r\n	Table 5 illustrates the work pressure factors for the respondents&#39; major source of stress. The three highest work pressure factors are work overload (28.6%), lack of job recognition (10.4%) and long working hours (9.5%).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	In the study, the prevalence of work stress among managers was 67.9% and executives were 68.1%. The prevalence of stress in this organization is higher compared to reported prevalence in other sectors. Fazil (2004) reported a prevalence of stress of 46.8% among factory workers in an electronic firm in Shah Alam, Selangor.&nbsp; Harmy et al (2001) reported a prevalence of stress of 36.8% among nurses working in the Intensive Ward in Hospital Universiti Sains Malaysia.</p>\r\n<p>\r\n	The study also showed that significant sociodemographic factors which had significant relationship were salary, number of children, and personal factors. The reasons of significant stress in relation to salary could be link to work stressors. For example, staff need to stay back to complete their work because of uncompleted task. The workers may feel that the salary earned may not be proportionate to the workload and long working hours put in by them.</p>\r\n<p>\r\n	The significant relationship between stress and number of children in this study is most likely due to the fact that having to support the children financially and they have to organize the heavy working schedules to allocate time to attend the children&#39;s need.</p>\r\n<p>\r\n	There is a significant relationship between stress and work stressors in this study. The heavy work overload could probably due to the need to carry out their jobs for supporting duties in the organization, meetings and presentation. Sometimes the time pressures and many datelines to meet making work too rigid. In order to meet the expectations, staff would have work extended hours. This finding is supported by a research done by Hasan (2002).&nbsp; The study evaluated job stress factors among heads of physical education organizations in Tehran University, Iran. The results indicate that a significant relationship between organizational job stress with pressure for work quality, job importance and time pressure.</p>\r\n<p>\r\n	This study is limited as it is a cross-sectional data, therefore time- causal relationship is less certain. The second limitation is the stress symptoms are self reported and thus information bias can occur.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In the study the prevalence of stress among the management staff in an international tobacco company has been determined. There was no significant difference in the level of stress between the executives and the managers in the company. The study highlights the need for intervention of stress.</p>\r\n', 1),
(393, 390, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Kimura&rsquo;s disease was first described in 1948 as an unusual granulation combined with hyperplastic changes in lymphoid tissue (Day 1995). Since then, series have been reported from China, Hong Kong, Singapore and Indonesia, all in patients of Mongoloid race.</p>\r\n<p>\r\n	The pathophysiology of KD may relate to a disturbance in the normal rate of production of eosinophils and IgE resulting in excessive elaboration of eosinophilo-trophic cytokines such as interleukin 4 (Day 1995).</p>\r\n<p>\r\n	KD is quite a rare condition. However, it might in fact be more prevalent than it seems for two reasons. First, most of the patients afflicted with this condition are asymptomatic and so they might go undiagnosed. Secondly, the lesions might have been misdiagnosed as other conditions; clinically it is often confused with parotid tumours with lymph node metastasis.</p>\r\n<p>\r\n	KD has the ability to mimic a number of benign inflammatory and neoplastic conditions of the head and neck including, Langerhans cell histiocytosis, Castleman&rsquo;s disease, Kikuchi&rsquo;s disease and follicular lymphoma. The most common physical manifestation is a slowly enlarging subcutaneous mass, often in the head and neck region involving the salivary glands with regional lymphadenopathy. It is usually associated with peripheral blood and tissue eosinophilia with markedly increased serum IgE levels (Irish 1994).&nbsp; FNAC and diagnosis is usually established on histopathological examination.</p>\r\n<p>\r\n	We describe a young boy who presented with a parotid mass due to KD, which was initially thought to be HL.</p>\r\n<p>\r\n	<strong>CASE REPORT </strong></p>\r\n<p>\r\n	A 16-year-old Malay boy presented with a painless swelling over the right parotid region, which was slowly increasing in size over the past one year. He had no constitutional symptoms. There was no previous history of tuberculosis (TB).&nbsp;&nbsp; Physical examination revealed a mass over the right parotid region measuring 6 x 6 cm that was firm and non-tender. There were enlarged lymph nodes over the right cervical region measuring 4 x 4 cm. There were no other significant physical findings.&nbsp; Full blood count revealed eosinophilia (eosinophil count 3.5 x 10<sup>9</sup>/l). The serum lactate dehydrogenase level and renal profile were within the normal range. Viral serology and TB work-up were negative.</p>\r\n<p>\r\n	A computed tomography (CT) of the neck showed an enhancing ill-defined diffuse mass and enlarged lymph nodes over the right mandibular region. The underlying subcutaneous tissue showed increased density suggestive of an inflammatory lesion. CT brain, thorax and abdomen did not show any abnormality. FNAC of the parotid mass was reported as HL, while trucut biopsy of the cervical lymph node showed reactive hyperplasia.</p>\r\n<p>\r\n	A right superficial parotidectomy and lymph node excision biopsy were performed, as isolated parotid lesion is an unusual presentation of HL. The lymph node tissues contained multiple lymphoid follicles and reactive germinal centres with marked perinodal fibrosis. Benign parotid tissues were noted surrounding the lymph nodes with periductal fibrosis, acinar atrophy and proliferation of endothelial venules. There were prominent eosino-philic infiltration and eosinophilic micro- abscesses within the nodal parenchyma and parotid tissue. Reed-Sternberg cells, giant cells and granulomas were not seen (Fig. 1A, 1B). The histopathological fea-tures were consistent with KD.</p>\r\n<p>\r\n	The patient was given oral prednisolone 60mg daily followed by a tapering dose over the next two weeks. At one-year follow-up (at the time of writing of this report), he remained disease free.</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	KD is an indolent, benign, but locally disfiguring disease.&nbsp; Its true importance lies in its ability to mimic a number of other benign inflammatory and neoplastic conditions of the head and neck. It is usually a localized process without sys-temic symptoms (Day 1995).</p>\r\n<p>\r\n	The most common causes of sub-acute or chronic cervical lymphadenitis, which should be considered in this case, are TB, Epstein Barr virus infection, toxoplasmosis, Kikuchi&rsquo;s disease and parotid tumours. The slowly enlarging mass and localised parotid involvement over a one year duration is rather unusual for HL. In addition, FNAC material alone is not conclusive for a definitive diagnosis of HL unless classical Reed-Sternberg cells are present. There were no radiological and microbiological evidence of TB and parotid involvement is unusual in TB. There was no laboratory evidence of viral or parasitic infection. The histological features which excluded Kikuchi&rsquo;s disease and malignancies.</p>\r\n<p>\r\n	Typical histological features in KD include preserved nodal architecture, florid germinal centre hyperplasia, eosinophilic infiltration, eosinophilic abscess, vascu-larization, necrosis of the germinal centres,&nbsp; proteinaceous&nbsp; deposition within germinal centres and fibrosis (Irish 1994). &nbsp;Specific histological features in HL include Reed-Sternberg cells and CD30<sup>+ </sup>mononuclear cells were absent in the present case. Other differences between these two conditions are a) KD has no potential for malignant transformation where else HL is a haematological malignancy, b) KD occur in younger adults between 20-40 years old while HL has a bimodal age distribution, c) KD is commoner in Asians while HL is commoner in Caucasians, d) KD is normally a localized process with no sys-temic symptoms but HL has constitutional symptoms in 40% of patients, and e) IgE is elevated in KD but not in HL.&nbsp;</p>\r\n<p>\r\n	There are three major therapeutic options for KD. Resection of the tumour mass may be effective in permanently eradicating the mass if the entire lesion can be removed, but recurrence is common (Motoi 1992). &nbsp;Systemic and intralesional corticosteroids have been shown to reduce the size of the lesion, but the tumour tends to recur when these drugs are discontinued (Motoi 1992).&nbsp; In selected patients, it may be advisable to take a conservative approach, treating only if the mass continues to grow or causes significant deformity. Local irradiation has also been shown to be effective in shrinking the lesions, but it is generally not advocated in younger patients (Motoi 1992).</p>\r\n<p>\r\n	In summary, KD is an indolent, benign disorder, but may easily be mistaken as a malignant lesion. With strict histologic criteria, a correct diagnosis can be achieved, especially when combined with pertinent clinical information and laboratory studies.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS </strong></p>\r\n<p>\r\n	We are grateful to Dr. Lee Suk Kum from the department of Pathology, Hospital Pulau Pinang for preparing the histo-pathology photographs.</p>\r\n', 1),
(394, 391, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Rocuronium bromide is a non-depolarizing neuromuscular blocking agent that may be used during &lsquo;rapid sequence induction&rsquo; (RSI) should suxamethonium be contra-indicated. However, its dose-dependent onset and duration of action occasionally limits its use. It has been reported that rocuronium is only comparable to suxa-methonium during RSI in 0.9-1.2 mg/kg (3-4x ED95) dosage, but its duration of action may be up to fifty minutes as opposed to thirty minutes when used at 0.6 mg/kg (Cooper et al 1992).</p>\r\n	<p>\r\n		The long duration of action of rocuronium at 0.9 mg/kg is an obvious disadvantage for short surgical procedures, besides adding to the overall anaesthetic costs. However, if rocuronium at the dosage of 0.6 mg/kg was used, 20-25% of patients had moving vocal cords on direct laryngo-scopy and there was some diaphragmatic response to intubation (Sparr et al 1996). This may be due to a slower onset of rocuronium at the laryngeal muscles and the diaphragm than at other muscles.</p>\r\n	<p>\r\n		The speed of onset of neuromuscular blockade depends in part on physiological factors such as cardiac output, circulation time and muscle perfusion. Studies using induction agents such as etomidate (Fuchs-Buder et al 1998)and ketamine (Hans et al 1999) which maintain cardiac output and blood pressure, have been shown to accelerate the onset of block and to improve intubating conditions when used in association with rocuronium.</p>\r\n	<p>\r\n		A study by Tanet al (2002) showed that when ephedrine 15 mg was added to propofol during intubation, the proportion of patients with excellent intubating conditions increased. However, this technique was associated with significant increase in mean arterial pressure and heart rate after induction. The improvement in excellent intubating conditions observed in this study was possibly due to improvement in cardiac output associated with the use of ephedrine. However, the effect of adding ephedrine at a lower dose of 70 &micro;g/kg on intubating conditions is not known.</p>\r\n	<p>\r\n		The use of fluid loading to treat hypo-tension during anaesthesia has been well established. Fluid preloading does not prevent hypotension associated with propofol, but it has been proposed to affect the cardiovascular response to propofol induction. Turneret al (1998) in their study have proposed that fluid preloading produces an increase in myocardial preload, leading to an increase in stroke volume and therefore, maintaining cardiac output at a lower heart rate without increasing blood pressure. Apart from adding ephedrine to propofol during induction, the crystalloid preloading such as normal saline prior to induction can possibly be used to maintain or improve cardiac output and hence, may influence the intubating conditions during induction with rocuronium.</p>\r\n	<p>\r\n		In order to investigate the effect of ephedrine and crystalloid on intubating conditions when rocuronium was used, this study was carried out. The objective of this study was to compare three different induction techniques which were propofol-placebo, propofol-ephedrine and propofol-crystalloid combinations on intubating conditions at 60 seconds using rocuronium 0.6 mg/kg.</p>\r\n	<p>\r\n		<strong>MATERIALS AND METHODS</strong></p>\r\n	<p>\r\n		This study was approved by the hospital ethics committee. Ninety patients with American Society of Anaesthesiologists (ASA) phy-sical status I or II, aged between 20-55 years and scheduled for elective surgery under general anaesthesia were included in this study. Subjects were excluded if they had a history of allergy to the study medication, hypertension, were on diuretics or vasoactive medication, were at increased risk of pulmonary aspiration and were on drugs known to interact with the neuromuscular junction. They were also excluded if we anticipated airway diffi-culties and their body mass index (BMI) were more than 30 kg/m<sup>2</sup>.</p>\r\n</div>\r\n<p>\r\n	Assignment to each group was concealed using sealed envelopes. On entering the study, patients were allocated randomly into one of three groups.</p>\r\n<table border="1" cellpadding="5" cellspacing="0" height="358" width="434">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width: 99px; height: 1px;">\r\n				<p align="center">\r\n					<strong>GROUP</strong></p>\r\n			</td>\r\n			<td style="width: 168px; height: 1px;">\r\n				<p align="center">\r\n					<strong>INDUCTION TECHNIQUE</strong></p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:99px;height:1px;">\r\n				<p>\r\n					Propofol-placebo (PP)</p>\r\n			</td>\r\n			<td style="width:168px;height:1px;">\r\n				<p>\r\n					Propofol (2.5mg/kg) and 1 ml of normal saline without ephedrine (placebo).</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:99px;height:1px;">\r\n				<p>\r\n					Propofol-ephedrine (PE)</p>\r\n			</td>\r\n			<td style="width:168px;height:1px;">\r\n				<p>\r\n					Propofol (2.5mg/kg) and 1 ml normal saline with ephedrine (70-&micro;g/kg).</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:99px;height:1px;">\r\n				<p>\r\n					Propofol-placebo-crystalloid (PC)</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:168px;height:1px;">\r\n				<p>\r\n					Ringer&rsquo;s lactate (12 ml/ kg) over the 10-15 min, propofol (2.5mg/kg) and 1 ml of normal saline without ephedrine (placebo).</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<div>\r\n	<p>\r\n		On arrival in the induction room, an 18-gauge cannula was inserted into a peri-pheral vein at the dorsum of the hand by the first anaesthetist who was not involved in assessing the intubating conditions score and charting the changes in heart rate and mean arterial pressure. Without the knowledge of the intubating anaes-thetist (second anaesthetist), the first anaesthetist prepared combinations of drug to be given according to the group assigned. Group PC was also given Ringer&rsquo;s lactate in the induction room.</p>\r\n	<p>\r\n		A non-invasive arterial pressure monitor, electrocardiogram (ECG) and pulse oxi-meter (SpO<sub>2</sub>) were attached, and baseline measurements were recorded. The sound of the pulse oximeter and ECG was then turned off and the monitoring screen was monitored only by the first anaesthetist. After preoxygenation of three minutes, anaesthesia was induced through an intravenous line with fentanyl 2 &micro;g/kg followed by one of the three combinations mentioned above, given over 30 seconds by a hand-held syringe followed imme-diately by rocuronium 0.6 mg/kg given over 5 seconds. Placebo or ephedrine, both prepared in 1 ml syringe were given just before propofol was administered to the patient.</p>\r\n	<p>\r\n		Sixty seconds after rocuronium was administered, laryngoscopy was attempted by the second anaesthetist who was blind to the ECG monitor and the drug given. During laryngoscopy and intubation of the trachea, the second anaesthetist per-forming the intubation assessed each patient for three different variables: jaw relaxation, vocal cords position and response to intubation. Assessment was based on the ease of laryngoscopy mani-fested by jaw relaxation, the position of the vocal cords and the reaction to tracheal intubation and cuff inflation as described by Cooper et al (1992). (Table 1). A total intubating score of 8-9 was considered excellent, 6-7 good, 3-5 poor and 0-2 bad. Excellent and good conditions were considered clinically acceptable, while poor and bad were considered clinically unacceptable.</p>\r\n	<p>\r\n		After confirming the placement of the endotracheal tube, anaesthesia was main-tained with sevoflurane, nitrous oxide and oxygen. Further recording of the heart rate and mean arterial blood pressure at 1, 2, 3, 4 and 5 min after tracheal intubation were done. Assessment of the intubating conditions and haemodynamic parameters were recorded by the second anaesthetist who was blinded to the study drug.</p>\r\n	<p>\r\n		Non-parametric data were analysed using the Kruskal&ndash;Wallis test, followed, where significant, by the Mann&ndash;Whitney <em>U</em>-test. For normally distributed data, the differences among groups were evaluated by ANOVA. A &lsquo;p&rsquo; value less than 0.05 was considered to be stastistically significant.</p>\r\n	<p>\r\n		<strong>RESULTS</strong></p>\r\n	<p>\r\n		The three groups were comparable in terms of age, gender, ASA physical status and BMI (table 2).</p>\r\n	<p>\r\n		Intubating conditions were clinically acceptable in all patients except in four patients from the propofol-placebo group who recorded poor intubating conditions.&nbsp; The percentage of patient with excellent intubating conditions was highest in Group PE (94%) followed by Group PC (81%) and lowest in Group PP (50%) (Fig 1).</p>\r\n	<p>\r\n		Jaw relaxation and vocal cords position scores however, did not differ significantly between the three groups (p&gt;0.05).</p>\r\n	<p>\r\n		The baseline value of mean arterial pressure (MAP) and heart rate (HR) did not differ among the three groups. There was no significant difference in the MAP of the three groups at 1, 2, 3, 4 and 5 minutes after intubation. However, there was a significant difference in the HR with the PE group recorded the highest increased in the heart rate at 1, 2, 3, 4 and 5 minutes after intubation (p&lt;0.01) (Table 3).</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		In rapid sequence induction, sodium thiopentone is commonly used as an induction agent. However in this study, we used propofol as the induction agent of choice as several studies (Dobson et al 1999, Barclay et al 1997) have shown that this agent is more superior to thiopentone in suppressing the laryngeal reflexes and has frequently been used for tracheal intubation with or without the use of muscle relaxants.</p>\r\n	<p>\r\n		Hypotension after induction of anaes-thesia with propofol is well recognized. Mulier et al (1991) in their study,have suggested a significant decrease in myo-cardial contractilityand systemic vascular resistance and that would account for the decrease in arterial blood pressure. Ephedrine can possibly be used to counteract the effect of myocardial depression by its action on beta-1 receptor which increases myocardial contractility. The use of this drug during induction has been postulated to increase cardiac output and tissue perfusion<strong>(</strong>Ezri et al 2003). This increase will accelerate the onset time of neuromuscular blockade by means of a faster delivery of neuromuscular-blocking drug to skeletal muscles.</p>\r\n	<div>\r\n		<p>\r\n			We have also demonstrated that the use of propofol-ephedrine and propofol-placebo-crystalloid combinations can im-prove intubating condition when rocuro-nium 0.6 mg/kg was used and the intuba-tions were performed at 60 seconds after induction. Intubating conditions were clinic-ally acceptable (good and excellent) in all patients in the study groups except in four patients in PP group, who have recorded poor intubating conditions. The proportion of excellent intubating conditions was significantly highest in the PE group followed by the PC group and lowest in the PP group.</p>\r\n		<p>\r\n			The improvement in intubating conditions observed in the PE group were most likely due to the effects of ephedrine. Since muscle blood flow was not measured in the present study, we can only speculate as to whether this variable was increased by ephedrine, and whether this was the mechanism for the observed improvement in the intubating conditions.In this study, we postulated that the improvement in intubating conditions were due to a faster delivery of rocuronium to the neuro-muscular junction. The finding of the improvement in the intubating conditions using propofol-ephedrine combination is also consistent with the findings by Tan et al<strong>(</strong>2002)<em>.</em></p>\r\n		<p>\r\n			Gamlin et al (1996) have shown that ephedrine reaches its peak effect in two to three&nbsp;minutes and exerts its effect in less than two&nbsp;minutes after its administration. Kim et al(2003) reported that ephedrine 70 &micro;g/&nbsp;kg given before the induction of anaesthesia improves tracheal intubating conditions at two&nbsp;minutes after vecuronium by increasing cardiac output without significant adverse haemodynamic effects. In this study, we used the same dose of ephedrine but the intubation was done at 60 seconds after administration of ephedrine.</p>\r\n		<p>\r\n			Turner et al (1998) in their study has proposed that fluid preloading produces an increase in myocardial preload, leading to an increase in stroke volume and therefore maintaining cardiac output at a lower heart rate without increasing blood pressure. Despite the drop in blood pressure, probably due to the drop in systemic vascular resistance, the cardiac output is well maintained. The delivery of muscle relaxant to the neuromuscular junction does not slow down and therefore, the fluid preloading has recorded better intubating condition as compared to placebo group observed in this study.</p>\r\n		<p>\r\n			In contrast with the study done by Tan et al (2002), we did not find any significant increase in the mean arterial pressure in the propofol-ephedrine group except from the increase in the heart rate associated with the use of ephedrine. The possible reason behind this observation might be due to a smaller dose of ephedrine used in this study. In this study, we used a dosage of 70 &micro;g/kg whereas in their study a dosage of 15 mg had been used which was about 3 times the dosage that had been used in this study.</p>\r\n		<p>\r\n			As the heart rate was significantly increased in the PE group, caution should be exercised in the use of this combination in patients with ischaemic heart disease. Further studies are needed to identify the ideal dose of ephedrine in this combination, along with quantitative measurement of the onset of neuromuscular blockade among the three combinations of induction agents.</p>\r\n		<p>\r\n			<strong>CONCLUSION</strong></p>\r\n		<p>\r\n			We conclude that induction with propofol-ephedrine and propofol-crystalloid provided significantly better intubating conditions than propofol alone, when rocuronium 0.6 mg/kg was used for intubation at 60 seconds.</p>\r\n	</div>\r\n</div>\r\n', 2);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(395, 392, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Cardiopulmonary resuscitation (CPR) is an essential skill for everyone especially those working in the medical field. It is composed of the recognition of signs and symptoms of impending or cardiac arrest (CA), heart attack, stroke and choking; chest compressions; and defibrillation with an automated external defibrillator (AED) (American Heart Association 2005). Regardless of the specialty of the physician, the skills and knowledge in performing these procedures are essential. Cardiac arrest is the leading cause of death in the US and also worldwide (Zheng et al. 1998). Annual incidence of CA is 0.55 per 1000 population (Vaillancort &amp; Stiel 2004).In Malaysia, the Information and Documentation System Unit of Ministry of Health Malaysia reported cardiovascular disease as among the top principle causes of death in the country.</p>\r\n	<p>\r\n		In the 2005 Consensus Conference by leading experts for the study on resuscitation, CPR was re-emphasized as the main initial intervention during any CA (Hazinski et al. 2005). Cardiopulmonary resuscitation should be done promptly and swiftly. It has been modified, making it easier to learn and apply. The complexities in the sequence, different ratios of chest compression to ventilation and the different age group ratios of the previous years have been removed. Now, there is only one compression to ventilation ratio that needs to be remembered by care providers, that is 30:2 (Hazinski et al. 2005).This simplicity was recommended so that more people will come forward and assist patients needing CPR.</p>\r\n	<p>\r\n		In the hospital, any of the ward patients may collapse. The first doctor who may be called to attend is the house-officer (HO). Senior medical officers (MO) or specialists usually come later. Therefore, the HO needs to immediately assess and recog-nize the situation and start CPR if needed. Hence, the knowledge and skill of the performer are essential and of utmost importance.</p>\r\n	<p>\r\n		CPR training is usually included in the medical curriculum in most medical schools. However, there are still some schools that fail to teach CPR to their students (Phillips &amp; Nolan 2001). In one survey in the UK, 4 out of 27 respondents failed to show that they provided CPR training in their curriculum (Phillips &amp; Nolan 2001).In Universiti Kebangsaan Malaysia (UKM), CPR is taught during the fourth year of the medical curriculum. Even though CPR is taught in medical school, the retention of the skills is a different issue. A meta-analysis demonstrated that professional skills can decay rapidly with-out practice or use (Arthur et al. 1998). This is worrisome. If it is true, then HOs are inadequately trained to perform competent CPR on patients. Therefore, immediate remedial measures need to be imple-mented. This study aimed to analyze knowledge, perception of skills and confi-dence level of performing CPR among HOs in our institution.</p>\r\n	<p>\r\n		<strong>METHODOLOGY</strong></p>\r\n	<p>\r\n		<strong>Study Design</strong></p>\r\n	<p>\r\n		This was a cross sectional study done on a sample of HOs working in Hospital Universiti Kebangsaan Malaysia (HUKM). Most were starting their medical career with 1-2 months work experience. They came from various clinical departments depending on their posting. The authors conducted this study during the second day of the orientation programme, planned out by the hospital human resource depart-ment. Each HO was given a serial number. A self-administered three-part question-naire was used to assess knowledge, perception of skills and confidence level of performing CPR among HOs. Thereafter, the completed questionnaires were collect-ed and analysed.</p>\r\n	<p>\r\n		<strong>Data Collection and Analysis</strong></p>\r\n	<p>\r\n		The questionnaire consisted of three parts. The first part dealt with demographic data of the HOs. The second part assessed their self-perception about their own CPR skills, confidence level in performing CPR and knowledge in performing CPR. The third and last part dealt with questions assessing their actual knowledge on the important aspects of CPR which included patient assessment, getting help, ABC&rsquo;s of CPR and use of AED. The total score was 11 points. A group of paramedics, medical officers and specialists working in the Emergency Department (ED) were asked to complete the questionnaire. By calcu-lating their mean score, we set the stan-dard that the score expected of a doctor competent in performing CPR was 9.</p>\r\n	<p>\r\n		After completing these questionnaires the HOs were given a short one and a half hours hands-on CPR course, which com-prised of CPR, use of AED, basic airway management and relief of Foreign Body Airway Obstruction (FBAO).</p>\r\n	<p>\r\n		Data was collected and tabulated in the SPSS 12.0 software where statistical analysis was done.</p>\r\n	<p>\r\n		<strong>RESULTS</strong></p>\r\n	<p>\r\n		There were 30 listed participants for the CPR orientation course, however only 27 of them attended. Out of the 27, one did not complete the questionnaires fully; therefore only 26 respondents were available for analysis.</p>\r\n	<p>\r\n		Twenty-two HOs were from a local university and four were from overseas universities. Their ages ranged between 24 to 37 years, with 22 (84.6%) of them, in the group of 24 to 26 years. Six were male while the rest were females with a male to female ratio of 1:3.3. Their rotational pos-tings included 12 in general surgery, 7 in internal medicine and 7 in obstetrics and gynaecology.</p>\r\n	<p>\r\n		Out of 26 subjects, 21 (80.8%) had done CPR training before while 5 (19.2%) had not. All except one had done their CPR training less than 2 years previously. From Table 1 we also can see that there were no differences in the percentage of those who had done CPR training and those who had not, among the groups that graduated from local public universities, local private universities and overseas universities.</p>\r\n	<p>\r\n		Their self-perception of knowledge in CPR was rated on a scale of 1 to 5 (1 was poor and 5 was excellent). Their meanscore was 2.2 (SD &plusmn; 0.7 / SE 0.1) [Figure 1]. In terms of their perception on theconfidence level in performing CPR, the mean score was 2.4 (SD&plusmn;0.9) (Figure 2).</p>\r\n	<p>\r\n		With regards to assessment on their actual CPR knowledge, the authors found that all subjects scored below 9/11. The highest recorded score was 8.7 and the lowest was 1.5 with a mean of 5.7/11 (SD&plusmn;1.8). On the aspect of &ldquo;correct hand placement during chest compression&rdquo;, 19 out of 26 (73.1%) were able to mark the correct spot for hand placement i.e. in the midline between the nipples on the lower half of the sternum.</p>\r\n	<p>\r\n		Automated external defibrillator paddle attachment was another aspect that was assessed. Four out of 26 HOs (15.4%) were able to properly place the sternal pad while 9 out of 26 HOs (34.6%) were able to locate the correct placement for the apex pad.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		The data represent a sample of HOs being posted every year in the Ministry of Health hospitals and University hospitals. Accor-ding to the registration book for new HOs reporting in, HUKM annually receives 25 to 30 HOs. They are assigned to three major departments, namely Internal Medicine, General Surgery and Obstetrics &amp; Gynae-cology. They undergo training under super-vision for a minimum of four months in each department before they are rotated to another department.</p>\r\n	<p>\r\n		The basis for conducting the study was to analyze the knowledge, perception of skills and confidence level of performing CPR among HOs. Since HOs will be the first line of medical encounter with sick patients in the wards, it is important for them to be clinically proficient and know-ledgeable in managing basic resuscitation. Resuscitation has been recorded even to the times of the Old Testament (Cooper &amp; Cooper 2006).</p>\r\n	<p>\r\n		&nbsp;It is the solid basis of the medical care doctors provide. Since then, the technique of basic resuscitation or CPR has been modified and refined (Cooper &amp; Cooper 2006).</p>\r\n	<p>\r\n		In terms of the actual place of gra-duation, this study population represents a somewhat similar situation to the rest of the country, where the majority of HOs come from local public universities. The next group is composed of those who come from local private universities and a third smaller group from overseas universities. In our study, we noticed that there are similarities among the HOs who had done basic life support (BLS) prior to their completion of their studies whether they are local graduates or from abroad. How-ever, the number was too small for any significant statistical comparison.</p>\r\n	<p>\r\n		In our study, we noted that knowledge on basic resuscitation skills or CPR, which is supposed to be a basic requirement for an HO, is lacking and insufficient for clinical work. The majority of the subjects per-ceived their knowledge and confidence level as inadequate with the mean score of 2.2 (SD&plusmn;0.7 / SE 0.1) and 2.4 (SD&plusmn;0.9) out of five respectively. They could be modest about their capabilities, however, even with the written test of knowledge it clearly shows that they did poorly. These two results correlate significantly as none of the subjects passed the written test. This is sad, since CPR skills were taught as a topic during undergraduate studies in UKM.&nbsp; In some countries, it is mandatory that before an HO enters the hospital, they must complete CPR training first(General Medical Council 1997). In fact, learning CPR has been made simple and less confusing since the year 2000, when the International Liaison Committee on Resus-citation (ILCOR) published <em>Resuscitation Guidelines 2000 for Cardiopulmonary Re-suscitation and Emergency Cardiac Care</em>(American Heart Association 2000). This guideline has made tremendous impact in terms of simplicity, similarity and necessity of BLS to be provided by all countries. Most countries have now adopted the 2005 ILCOR CPR guidelines.</p>\r\n	<p>\r\n		These findings show that CPR skills are not emphasized enough during medical student times.&nbsp; Another probability is that the medical student themselves do not bother to learn CPR because medicine is so broad and has become more techno-logy-based. Thus, most of the students seem to feel that CPR is too basic compared to all the other information they have to know.</p>\r\n	<p>\r\n		These results are alarming. Where do we proceed from here? What do we need to do? If this trend persists, it is high time that we review our medical curriculum. The curriculum may need some adjustment on the subject of CPR. The Emergency medicine posting should be a requirement for medical students. The delivery method of this topic (CPR) also needs to be reviewed so that it attains more retention of skills and practical knowledge rather than theoretical information only.</p>\r\n	<p>\r\n		The study found that 24 out of 26 of the subjects did their CPR course 2 years earlier. As what was suggested by many institutions like the American Heart Asso-ciation (AHA), European Resuscitative Council (ERC), Australian Resuscitative Council etc. the validity of a CPR certificate is about 2 years (Cummins et al. 1997).In this case, the majority of the subjects still had a valid CPR certificate. However, since most did badly, we suggest that the timing of the refresher course needs to be re-viewed. The retention of the skills learned deteriorates quite rapidly if the skills are not practiced (Wollard et al.2004 &amp; Makker et al. 1995).</p>\r\n	<p>\r\n		The authors feel that to complete only one CPR course during the whole five or six years term of medical study may be inadequate. We would suggest that a CPR course be conducted more than once. If possible the final course should be done just before they leave the medical school. Although most medical schools are claiming the issue of not enough man-power to run the course, the refresher course need not be the same as the initial course. A shorter course with more em-phasis being put on the simulation tech-nique and scenario-based learning can achieve similar results (Wayne et al. 2006).</p>\r\n	<p>\r\n		This study has its own limitations. The sample size is small to be reflective of the whole HO population in the country. The authors feel that a similar study should be repeated on a larger study population to document the actual representation of HOs and to validate the actual extent of this identified problem. From this study, we have identified an important issue and insight of the medical education curriculum on CPR training.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		From this study, the authors conclude that the HO&rsquo;s knowledge, perception of skills and confidence levels in performing CPR is insufficient and needs improvement. We recommend that the faculty should review the medical curriculum on CPR teaching and training needs. Strong emphasis needs to be placed on CPR training analyzing properly the method of delivery, timing and frequencies of the training course and refresher courses.</p>\r\n</div>\r\n', 1),
(396, 393, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Home care Nursing Program at HUKM was started in 1998. Since its inception, the patients and type of cases managed were those related to medical and surgical problems. They included amongst others; wound breakdown, stoma care, removal of stitches, diabetic ulcer, bronchial asthma and post CABG patients. The most common cases referred to the HCNP were Cerebral Vascular Accident (CVA) or diseases affecting the nervous system. In fact, at the beginning of the program 99% of the cases referred to HCNP were CVA cases. This could be due to awareness of the neurologist about the existence of the HCNP at HUKM. For this reason the researcher had chosen CVA patients as participants of this study and to measure effectiveness of the HCNP. The objectives of this study are: i) to determine the extent of disability of stroke patients who were referred to HCNP at HUKM. ii) to determine the relationship between services required from the HCNP and the extent of disabilities of stroke patients. iii) to compare the stroke patients who had been referred to the HCNP to those who had not with regards the ability to perform the daily living activities.</p>\r\n<p>\r\n	The purpose of home care nursing program was to provide service to post hospitalized patients at home. As a result of this program the patient could be sent home earlier (Alliance for academic Home Care 2001). The function of home care nurses was to teach the patient and care giver at home (Green 1998). The effectiveness of home care nursing had been proven on all types of cases. Patients with chronic obstructive pulmonary disease improved in cycle workload dyspnea scores significantly better in home care rehabilitation program compared to hospital-based rehabilitation program (Strijbos, Postma, van Altena, Gimeno &amp; Koeter 1999). Randomised Controlled Trial of rehabilitation at home after stroke in Southwest Stockholm by Holmqvist et al (1998) involving home and routine rehabilitation showed positive effects of the home rehabilitation group in social activity, activities of daily living (ADL), motor capacity, manual dexterity and walking. In another study conducted by Mayo et al (2000), there was significant beneficial impact of home intervention on ADL and reintegration to normal life. Duncan et al (1998) suggested that any structured exercise program should be planned for post stroke patients to ensure effectiveness of their rehabilitation. This was because results of their study showed that the experimental group tended to improve more than the control group in motor function, and upper extremities, though there was no significant difference in the scores of hand function.</p>\r\n<p>\r\n	<strong>METHODOLOGY</strong></p>\r\n<p>\r\n	A retrospective matched-paired design was used to evaluate the effectiveness of the HCNP on activities of daily living of post stroke patients. The objectives of this study are; i) to determine the number of stroke cases and the extent of disability using Modified Barthel Index (Shah at al 1989) who were referred to the HCNP at HUKM, ii) to compare the stroke patients who had been referred to HCNP and those who have not with regards their ability to perform the activities of daily living. The study was conducted at HUKM, one of the teaching hospitals in Malaysia. The hos-pital is run by Clinical Specialists who are academic staffs of the Faculty of Medicine, UKM. The hospital functions as a referral centre in the country. As such, HUKM is both a teaching hospital and a tertiary referral centre for therapy and second opinion. The HCNP was started at HUKM for the purpose of fulfilling its social respon-sibility as well as to relieve the congestion in the hospital. The management of HUKM believe that involvement of members of the community in taking care of their family members would help in the recovery pro-cess of patients. The study sample in-cluded all stroke patients who were ad-mitted during 1998-2000 and consisted of two groups; the HCNP (n=43), and Non-HCNP group (n=73). Stroke patients who were not referred to HCNP and lived outside the coverage areas, plus patient who had a history of mental problems were excluded. The Modified Barthel Index when tested by many authors, showed internal consistency coefficients of 0.87 to 0.92 (Shah et al. 1989), 0.99 (Kucukdeveci et al. 2000) and 0.90 to 0.93 (Nazzalm et al. 2001).</p>\r\n<p>\r\n	The data collection process went through a few stages, firstly the research scrutinised the list of stroke patients who had been admitted from August 1998 to December 2000. The patients listed in the data were categorised under the Inter-national Code of Disease (ICD) 9 code 436 (those patients admitted from January 1998 to May 1999) and ICD 10 code 164 (those patients admitted from June 1999 to December 2000). With the help of ward staffs the researcher managed to trace important information about patients such as permanent address, registration num-ber, name, age, contact number etc. The Health Assistant from the HCNP unit who was not involved in giving care to the stroke patients was given the task of contacting the participants via phone. If the participant was reluctant to participate, their name was excluded. All the partici-pants contacted volunteered to participate. Next, the process of obtaining the partici-pant&rsquo;s particulars before entry into the questionnaire forms was done. The re-searcher obtained demographic data of patients, and a Consultant scored the ADL during admission based on the physical assessment during admission obtained from their medical records. The process of filling up the questionnaires was done at the record office. Finally, the participants are interviewed by the researcher with the help of a Research Assistant. Each inter-view took 20 to 30 minutes for every participant.</p>\r\n<p>\r\n	<strong>RESULT</strong></p>\r\n<p>\r\n	Out of 43 stroke patients in the HCNP group, only 35 (81.3%) were included in this study, because six (13.5%) had died and two (4.6%) were transferred to another locality and could not be traced. Six patients who died were very ill. In the Non-HCNP group, of the 74, only 34 were comparable with the HCNP group and were included in the study. The total number of the stroke patients admitted to HUKM during the period of study for both groups were inaccurate as records were<br clear="all" />\r\n	incomplete.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The respondents in this study were stroke patients and there was no significant differ-rence in terms of age (t=3.057, p=0.85), race (Cramer&rsquo;s V=0.267, <em>p</em>=0.178), sex (<em>x<sup>2</sup></em>=0.013, <em>p</em>=0.911), time of admission (Cramer&rsquo;s V=0.295, p=0.647), and medical problems (Cramer&rsquo;s V=0.220, <em>p</em>=0.188). The activities of daily living on admission was statistically not significant. The level of dependency was classified into five categories; i) totally dependent on care giver with maximum help needed by the post stroke patients was 27 hours per week. There were 28 (80%) out of 35 from the HCNP group who fell into this category. In the Non-HCNP group 22 (64.7%) out of 34 participants showed total dependence on the caregiver. Even though it was statis-tically not significant, the percentage of the HCNP group was higher (80% vs 64.7%). The main reason why the Non-HCNP group did not refer to the HCNP unit for post hospitalised care was because they were living outside the coverage areas within 15 kilometers radius to HUKM. ii) The second category was greatly dependent on the caregiver when their ADL scores was 25-49 involving six (17.%) respondents from the HCNP group, and 10 (29.4%) from the Non HCNP group. The level of dependency required 23 hours of help per week (maximum) from the caregivers. iii) The third category was &lsquo;moderately&rsquo; dependent on caregiver. Only one (2.8%) in the HCNP group and two (5.9%) of the Non-HCNP group were in this study and they needed a maximum 20 hours of help per week. Only the above three categories participated in the study, whereas the other two categories (mildly &amp; minimally) were not referred to the HCNP.</p>\r\n<p>\r\n	&nbsp;The assessment results of ADL of post stroke patients showed that out of 28 (80%) patients, nine (25%) of the stroke patients were still totally dependent on caregivers (ADL score 0 - 24), three (8.6%) show slight improvement from being totally dependent to severely dependent (ADL score 25 - 49). Three (8.6%) were moderately dependent on caregivers (ADL score 50 - 74), seven were mildly dependent and six (17.1%) improved and were minimally dependent on the caregiver</p>\r\n<div>\r\n	<p>\r\n		(ADL 91 - 99). There were 6 (17.1%) patients with ADL scores between 25 - 49 and were severely dependent on care-givers at the time of admission. Out of these, one (2.8%) became totally depend-ent on caregivers (ADL score from 25 - 75 to 0 - 24), two (5.7%) were mildly depend-ent and three (8.6%) were minimally de-pendent on caregivers.</p>\r\n	<p>\r\n		In the Non-HCNP group, out of a total of 22 patients, 10 (29.4%) of the stroke patients were still totally dependent on caregivers (ADL score 0 - 24), and four (11.8%) were slightly better; i.e. severely and moderately dependent on the care-givers (5.9% for each level). There were five (14.7%) who were mildly dependent on caregivers. There were 10 patients with ADL scores of 25 - 49 at the time of admission. One (2.9%) mildly dependent on caregiver and nine (26.5%) were minimally dependent on caregivers.&nbsp; Two (5.9%) of the patients in this group have ADL score of 50 &ndash; 74 at the time of admission. These two patients have minimal level of dependency post stroke; i.e. (ADL score 91 &ndash; 99) (Table 5).</p>\r\n	<p>\r\n		In summary, those patients who were mildly and minimally dependent on caregivers with ADL scores of 75 &ndash; 90 and 91 &ndash; 99 respectively, the patients were considered fully rehabilitated. The mean score in the HCNP group was 3.20 and in the Non-HCNP 3.35.</p>\r\n	<p>\r\n		The independent (t=0.382, df 67 and p=0.704) showed no significant difference in terms of the level of dependency on caregivers post stroke for both groups.</p>\r\n	<p>\r\n		The alternative treatments were still popular and had been practised in Malaysia, because out of 35 patients from HCNP 25 (71.4%) and 25 (73.5%) from Non-HCNP practised it. Massage is the most popular choice taken either used as a single treatment or combined with acupuncture, herbal medicine, and refle-xology. The results of this study showed that ADL scores for patients using alter-native treatments were better irrespective of whether they were from the HCNP or the Non-HCNP groups. Studies in the USA showed the positive effects of massage on the body systems (Travell &amp; Simons 1983). Massage helped to decrease muscle tension and simultaneously repair damaged muscles after repeated treatments. The positive effects of refle-xology in rehabilitation of stroke patients had also been reported (Kunz &amp; Kunz, 2001). A few studies showed the positive effects of acupuncture on stroke rehabi-litation (Shiflett 2001; Asplund 2001; Johansson 1993) with significant difference obtained for both groups when activities of daily living score on admission and activities of daily living of post stroke participants who sought alternative treat-ments and those who did not were compared. For the HCNP group, the resultshowed t =-9.654, (df=68, p=0.000), and for the Non-HCNP the result showed t=-9.153, (df=68, p=0.000). There was a significant difference between activities of daily living on admission and activities of daily living post stroke for both groups.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		In this study, a comparison was made between the HCNP and Non-HCNP stroke patients. The results showed no significant difference between the two groups. However, from the results of the study it had identified one weakness, i.e. lack of knowledge about the concept of the HCNP by the service nurses. For example, there was one patient from a nursing home who was accepted as a client. This should not have happened because based on the original concept of the HCNP the rehabilitation of the patients were under the responsibilities of the family members/ caregivers. In this patient she had no rela-tive for the nurses to transfer the complex skills to assist her in the process of rehabi-litation. Clients were still dependent on the nurses in performing the complex skills. As such, they were not in a position to take care of their own health. The reason was that the Nurse Manager did not give the nurses proper instructions or briefing on the concept of home knowledge or skills to the clients. With the clients&rsquo; dependence on the nurses it had definitely prolonged the process of rehabilitation. This could lead to increased complications and frequent re-admissions. In addition, the nurses lacked the confidence to reject cases which were not for home care nursing. Nine patients out of 35, who were totally dependent on the caregivers, were referred to the HCNP only for the purpose of changing the Ryle&rsquo;s tube. These types of cases should be rejected by nurses or managed more holistically.</p>\r\n	<p>\r\n		<strong>ACKNOWLEDGEMENTS</strong></p>\r\n	<p>\r\n		I would like to express my warmest thanks to Associate Professor Nik Safiah, Asso-ciate Professor Tony Barnett, Associate Professor Khadijah, Associate Professor Dr Hamidon, Dr Rizal, Dr Safari, Head Nurse Kamsiah, and many others who were involved in the study. But most important of all, I would like to thank the participants who made this study possible.</p>\r\n</div>\r\n', 1),
(397, 394, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Cervical cancer is one of the most common cancers in Malaysia. It is the second commonest cancer for women. According to the Malaysian National Cancer Registry, the percentage when compared with the rest of the cancer was 12.9% (Lim et al 2003). One of the most important cervical cancer risk factors is human papillomavirus (HPV) infection (Munoz et al. 2003). Up to now, more than 200 HPV types have been identified (Bernard 2005). The International Agency for Research on Cancer (IARC) studies has provided generic and type-specific risk estimates for HPV. They conclude that there is a strong association between HPV and cervical cancer develop-ment and the risk of developing cancer depends on the HPV type (Munoz et al. 2003). However, HPV infection alone is not sufficient for cervical carcinogenesis, and attention has been focused on other factors important to this process. The development and progression of cervical cancer are likely to be associated with loss of growth suppression (uncontrolled proliferation), increased cell growth rates, and angioge-nesis (Araujo Souza et al. 2003, Tjalma et al. 2001, Stanley 2001).</p>\r\n<p>\r\n	The p53 gene is located on chromosome 17p13.1 and it functions as cell-cycle arrest and apoptosis in response to DNA damage. It is the most common target for genetic alteration in human tumours. Homozygous loss of p53 gene activity can occur in almost every type of cancer, such as carcinomas of the lung, colon and breast. Normal p53 protein has a very short half life and thus the protein level is too low to be identified immunohistochemically. In contrast, mutant p53 proteins have a longer half-life (Finlay et al. 1988)and can be easily detected by immunohistochemi-cal methods. Overexpression of p53 pro-tein has been identified immunohistochemi-cally in a variety of tumours.</p>\r\n<p>\r\n	The p53 gene is one of the most important targets of the HPV E6 gene. It was found that E6 protein of high-risk HPV, but not of low-risk HPV, could interact with p53 in vitro systems. In addition to the ability to stimulate p53 degradation, E6 protein inhibits several functions of wild-type p53 and it competes with it for the most important p53 protein functions inclu-ding a major role in the suppression of malignant growth. It was also shown that E6 increased the level of mutagenesis and genomic instability (Storey et al. 1998, Kisseljov et al. 2000). The resultant loss of wild-type p53 increases cellular genomic instability after DNA damage.</p>\r\n<p>\r\n	The aim of the present study was to determine and compare the expressions of p53 protein in CIN and cervical cancers, which includes squamous cell carcinoma, adenocarcinoma and adenosquamous<br clear="all" />\r\n	carcinoma.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Study Design</em></p>\r\n<p>\r\n	This is a retrospective study on cases diagnosed as CIN, squamous cell carcinoma (SCC), adenocarcinoma (AC) and adenosquamous carcinoma (ASC) obtained from the histopathology records of the Department of Pathology, Hospital Uni-versiti Kebangsaan Malaysia (HUKM), for the past seven years from January 1, 1994 to December 31, 2000. The total number of cases was 100. There were 21 cases of CIN1, 8 cases of CIN2, 25 cases of CIN3, 36 cases of SCC, 7 cases of AC and 3 cases of ASC.</p>\r\n<p>\r\n	<em>Antibody and immunohistochemistry</em></p>\r\n<p>\r\n	Three-micron thick sections were cut from the paraffin blocks and mounted on sialinized slides. Sections were then depa-raffinized with 2 changes of xylene and rehydrated with four changes of alcohol in decreasing concentrations at 3 minutes each and then washed in water. The sections were then treated with target retrieval solution (Dako Corporation Denmark) in 1:10 dilution for 20 minutes in a water bath at 97<sup>o</sup>C, followed by cooling for 20 minutes in room temperature. This is followed by staining using DAKO LSAB+ Kit peroxidase (Dako Corporation Den-mark). The slides were treated with 3% hydrogen peroxidase for 10 minutes to block endogenous peroxidase activity. Then, the slides were washed in Tris-buffered saline (TBS) for 2 changes at 5 minutes each. They were then incubated in monoclonal antibody p53 protein (D07, Dako Corporation Denmark) at a dilution of 1:500, for 30 minutes. Streptavidin and diaminobenzidine (DAB) were added. Finally, the slides were counterstained with haemotoxylin and eosin. For all cases, the same technical personnel performed the immunohistochemical stainings. Sections of breast cancer were used as positive control.</p>\r\n<p>\r\n	<em>Interpretation of result</em></p>\r\n<p>\r\n	All slides were examined under light micro-scopy. Distinct nuclear staining was re-garded as p53 positivity. 100 cells were evaluated in representative high-power fields, to obtain the percentage of cell positivity. The pathologists were blinded to the clinical diagnoses and origin of the samples. The degree of nuclear staining was graded depending on the percentage of cells stained; grade 0 samples with no positivity (negative); grade 1 when less than 10% of cells showing positivity (mild expression), grade 2 with 11% to 50% expression (moderate expression), and grade 3 with&nbsp; greater than 50% expression (intense expression).</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	The difference in the degree of p53 protein staining between CIN (pre-malignant) and carcinoma (malignant) was assessed using the Pearson chi-square test. The percent-age of reactivity in the different individual types of cervical neoplasms, i.e., CIN1, CIN2, CIN3, SCC, AC and ASC were categorical variables. P value was calcula-ted by SPSS program version 12.0 (SPSS Inc., Chicago, IL, USA). Any p value &lt; 0.05 was considered to be statistically signify-cant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Clinical Presentation</em></p>\r\n<p>\r\n	The age of clinical presentation of patients with cervical neoplasm (CIN and invasive carcinoma) ranged from 20 to 72 (43.8) years. Patients with cervical carcinoma (32 to 72, mean 50.3 years) were older than those with CIN (20 to 58, mean 37.8 years); four of our patients who had CIN were younger than aged 30 years. The distribution of cervical neoplasm in different ethnic groups in Malaysia is 53% Chinese, 37% Malays and 10% Indian.</p>\r\n<p>\r\n	<em>p53 Expression</em></p>\r\n<p>\r\n	Of the 100 samples obtained, 54 cases were pre-malignant lesions (21-CIN1, 8-CIN2, 25-CIN3), 46 cases were malignant (36-SCC, 7-AC and 3-ASC. Positive staining for p53 overexpression was localized in the nuclei of carcinoma cells (Figure 1). The relationship between the percentage of cells with p53 expression and histological diagnosis is summarized in Table 1.</p>\r\n<p>\r\n	Thirty six of the 54 pre-malignant cases (66.7%) were positive for p53 protein, in contrast to the malignant cases in which, 40 of the 46 cases (87.0%) were positive. When the intensity of staining is evaluated, grade 3 or intense expression of p53 protein was observed in 30/46 (65.2%) of the malignant cases, while only 8/54 (14.8%) of the pre-malignant cases were immunoreactive. The majority of CIN showed negative to grade 1 (mild expression) (29/54, 53.7%) immuno-staining. In contrast, 84.8% (39/46) of the invasive carcinoma showed grade 2 to 3 (moderate to intense) immunostaining. The difference in the intensity of p53 protein immunoexpression was statistically signi-ficant (p value &lt;0.05). (Table 1)</p>\r\n<p>\r\n	In the pre-malignant lesions, grade 3 or intense expression of p53 was noted to be higher in CIN3 (6/25, 24%) when compared to CIN1 (1/21, 5%) and CIN2 (1/8, 12.5%) (Figure 2). However, 7 of the 25 CIN3 cases were found to be negative for p53 expression. In malignant cervical lesions, SCC (26/36, 72.2%) was observed to have the highest percentage of cases with in-tense p53 expression, this is followed by AC (3/7, 42.9%) and ASC (1/3, 33.3%) (Figure 3).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	p53 overexpression is linked with the control of cell growth, cell cycle, and apoptosis (Levine 1992, Levine et al. 1991, Greenblatt et al. 1994). Wild-type p53 protein usually resides in normal cell nuclei. This protein is unstable and has a half-life of only 20&ndash;30 min, while the mutant p53 protein is more stable and has a pro-longed half-life, resulting in detectable im-munohistochemical staining (Levine 1992, Greenblatt et al. 1994). In uterine cervical carcinoma, the detection rate of p53 overexpression by immunohistochemistry has been reported to range from 8 to 74%, (Huang et al. 2001, Avall-Lundqvist et al. 1997, Chen et al. 2000, Oka et al. 1993, Brenna et al. 2002, Helland et al. 1993, Cavuslu et al. 1997, Dejonge et al. 1999, &nbsp;ter Harmselet al. 1998, Holm et al. 1993) and some reports have suggested that p53 overexpression is important as a prog-nostic marker (Huang et al. 2001, Avall-Lundqvist et al. 1997, Chen et al. 2000, Oka et al. 1993, Holm et al. 1993).</p>\r\n<p>\r\n	In the present study, p53 overexpression was detected in 87% of all invasive cervical carcinoma cases (40/46). Other investiga-tors have reported similar findings (Ikuta et al. 2005). Our study did not show a significant difference in p53 expression</p>\r\n<p>\r\n	between the degrees of CIN, although a slight increased in the percentage of cases with intense p53 expression was observed in CIN3. On the contrary, when pre-malignant lesions were compared with malignant lesions, there is a significant difference. This suggests that p53 altera-tion is not an early event in the pathogen-nesis of cervical cancer.</p>\r\n<p>\r\n	AC (Suzuki et al. 2004) and ASC are thought to carry a less favourable prog-nosis compared to SCC, therefore p53 expression would be expected to be greater in the former. However, p53 expression was conversely noted to be greater in SCC. This finding does not reflect the true population because there were only 7 cases of AC available for evaluation. Many reports have demon-strated the correlation between p53 gene mutation and unfavorable outcome in various human cancers, including lung (Mitsudomi et al. 2000), breast (Rahko et al. 2003), bladder (Lorenzo-Romero et al. 2003), and cervical (Oka et al. 2000)cancer. Previous reports have shown that p53 overexpression has a statistically significant correlation with poor outcome of cervical SCC (Oka et al. 2000) and AC (Suzuki et al. 2004).</p>\r\n<p>\r\n	Epidemiological studies have shown that HPV infection, especially high-risk HPV such as HPV types 16 and 18, is involved in the carcinogenesis of uterine cervical carcinoma (Walboomers et al. 1999, Zur Hauzen 2002, Munoz et al. 2003). It is known that high-risk HPV produces E6 oncoprotein, which binds to wild-type p53 protein to inactivate and degrade tumor suppression (Graflund et al. 2002, Kedzia et al. 2002, Crook et al. 1992, Werness et al. 1990, Akasofu et al. 1995). It is therefore postulated that loss of p53 function due either to binding of HPV coding proteins or mutations of the p53 gene is an important event in the patho-genesis of cervical carcinomas (Akasofu et al. 1995). Further investigations to deter-mine the HPV status in our population is crucial.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, the expression of p53 is significantly higher in malignant cervical neoplasms than in pre-malignant cervical lesions, suggesting that p53 overex-pression is not an early phenomenon in the pathogenesis of cervical cancer. It is also shown to be slightly higher in percentage in CIN2 and CIN3 (high-grade lesion) com-pared to CIN1 (low-grade lesion). This may be due to the fact that high grade lesions are more likely to have high risk oncogenic human papillomavirus infection as com-pared to low grade lesions.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(400, 397, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The working environment of Intensive Care Unit (ICU) is a constant source of stress for nurses working there. Critical care nurses practice in a complex assessment, high intensity therapies and continuous nursing vigilance (Norbeck 1985). Nurses con-stantly experience a stressful environment because of the complex nature of patient&rsquo;s health problems requiring an extensive use of very sophisticated technology (Robinson &amp; Lewis 1990). Nurses not only have to cope with the sophisticated technology but also regularly face ethical dilemmas con-cerning issues of patient care manage-ment. The changes in Health care delivery has also created new nursing roles and responsibilities and has also contributed to the source of stress (Erlen &amp; Sereika 1997). Stress occurs when nurses try to manage patient&rsquo;s nursing care within the scope of nursing. Stress also occurs when there is a constant desire to achieve only the best.</p>\r\n<p>\r\n	<strong>Types of work stressors</strong></p>\r\n<p>\r\n	Several researchers have described ICU as a stressful environment (Robinson &amp; Lewis 1990) and Youngner et al (1979) found that ICU environment itself creates stress for nurses. Studies have also shown that high level of stress can affect the com-petence and job performance of nurses (Taromina 2000). Hay &amp; Oken (1972) identified the ICU environment and the psychological impact of working in an ICU as two factors contributing to the stress experienced by ICU nurses. However, a study done in Netherlands on 521 nurses did not agree with the idea that the work environment in ICU is more stressful than a non ICU (Boumans &amp; Landeweerd 1994).</p>\r\n<p>\r\n	&nbsp;&nbsp; Some researchers have indicated issues related to death and dying of patients nursed closely with the hope of recovering as fully stressful for ICU nurses. Decision making is another factor that is a concern for ICU nurses. Previous research has also indicated that the source of stress among ICU nurses is more organizational in na-ture. Organizational stressors could be physical, psychological and social in nature (Duquette et al 1994). Nurses working in environments where verbal and physical aggression is prevalent are more likely to experience the effects of stress. The other determinants causing the stress in ICU nurses could be due to increased job de-mand, nursing shortage (Janseen et al 1999), overload of work, lack of social support from colleagues (Balker et al,2000), a lot of paperwork and high expectations from the superior, organiza-tion and next of kin of patients.</p>\r\n<p>\r\n	<strong>Effect of work stressors</strong></p>\r\n<p>\r\n	According to Dunham (1984), response to stress can be divided into three categories i.e mental, physical and emotional. Emo-tional and mental responses to stress has been linked to outbursts of anger, unne-cessary worries and frequent mood changes whereas physical stress is asso-ciated with cardiac disorders, ulcer and skin rashes. Stress has also been found to be associated with mood changes which include tension, anxiety, fatigue and de-presssion (Cox &amp; Ferguson 1991). Effect of burnout can range from a degree of dysfunction to exhaustion and loss of con-trol (Tavares 1994).&nbsp; Previous studies on stress among nurses have identified posi-tive relationships between work stress, mental distress (Tyler &amp; Ashway 1992) and high incidences of stress. Little is known about the prevalence of stress and the factors influencing stress among the nurses in ICU in HUKM. This study thus aims to investigate the prevalence of stress among the critical care nurses, factors influencing the stress and to examine the relationship between stressors and nurses.</p>\r\n<p>\r\n	<strong>METHODS</strong></p>\r\n<p>\r\n	A total of 70 critical care nurses working in ICU, Hospital Universiti Kebangsaan Ma-laysia (HUKM), were recruited in this cross-sectional study. The ICU of HUKM was chosen because of it being a referral center and a teaching hospital, its utilization of so-phisticated technology in the management of critical patients and also partly because patients here are managed by trained spe-cialists and anaesthetists. Questionnaires adapted from Cole (1992) were distributed to all nurses who were recruited, using a random universal method which means all nurses working in ICU, who meet the in-clusive criteria were recruited into this study. The inclusive criteria used in the recruitment of the samples are that they must be registered with the Nursing Board of Malaysia. The questionnaire was tested for validity and reliability through a pilot study conducted on 20 nurses working in the Intensive care unit of Hospital Tengku Ampuan Rahimah Klang. The results of the pilot study were not included in the actual study. Questionnaires consisted of four sections: section A on socio-demographic data, section B consisted of 25 stress in-ventory items divided into three subtopics: physical&nbsp; environment (9 items), psycholo-gical environment (10 items) and nurses responses towards stress in the past 12 months (6 items), section C are question-naires on the study variables influencing stress and section D on coping mechanism of stress. For the questions in relation to responses towards stress, nurses are just required to answer &lsquo;Yes&rsquo; or &lsquo;No&rsquo;.</p>\r\n<p>\r\n	Data was coded and entered into SPSS version 10.0 (Statistical Package for Social Sciences) for descriptive analysis. Data were described by frequencies and percen-tages. A Chi square was performed to in-vestigate the relationship between factors influencing stress and socio-demographic data.&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<strong>Description of the samples</strong></p>\r\n<p>\r\n	Out of the 70 questionnaires distributed, only 67 sets were returned with full infor-mation whereas 3 sets were returned emp-ty. More than 70% of the nurses were 22-40 years old. Only 13% were between the age of 25-27 and an equal number of nurses between 28-30 years and over the age of 31. All the nurses had completed their secondary education and were regis-tered nurses. Majority of the nurses (80%) have a short working experience of less than three years. Out of the 67 nurses, only 10 (14.3%) nurses had undergone the post basic course in ICU. Based on the short working experience and not having a post basic course (87.5%), nurses may not have a strong foundation and knowledge in rela-tion to critical care nursing. In relation to marital status, 57 (81.4%) of the res-pondents were married and 16 (22.9%) were found to be living with families. Living on their own accounted for 20% and living with friends 57.1%.</p>\r\n<p>\r\n	<strong>Prevalence of stress </strong></p>\r\n<p>\r\n	All (100%) of the nurses said they had experienced physical stress, psychological stress and changes in behaviour and indicated the symptoms of stress they ex-perienced in the past 12 months through the stress inventory.</p>\r\n<p>\r\n	<strong>Stress inventory</strong></p>\r\n<p>\r\n	From the total number of 67 nurses, 66 (98.6%) experienced episodes of head-ache followed by abdominal pain (60%) and chest pain (57.1%). Sixty-five (97.1%) nurses were also taken ill with viral infection, experienced sleeping problems (84.3%) and weight loss (65.7%). However, loss of libido was experienced by only a minority (5.7%) of nurses (Table 1).</p>\r\n<p>\r\n	<strong>Nursing a critically ill patient</strong></p>\r\n<p>\r\n	A total of 50 (71.4%) nurses indicated they feel stressful when nursing a critically ill patient compared to 53 (75.7%) when nursing patients who require resuscitation.</p>\r\n<p>\r\n	<strong>Nurse-patient ratio</strong></p>\r\n<p>\r\n	Nurse-patient ratio was identified as a stress factor by 100 percent of the nurses. However, nurses who were given only one patient to care finds it less stressful compared to nursing two to three patients.</p>\r\n<p>\r\n	<strong>Environmental factor</strong></p>\r\n<p>\r\n	The majority (67.1%) indicated that being in an environment with sophisticated machines as very stressful to handle as operating and fixing the machines requires a lot of knowledge and patience.</p>\r\n<div>\r\n	<p>\r\n		<strong>Interpersonal relationship</strong></p>\r\n	<p>\r\n		Thirty-one (44.9%) nurses finds it uncom-fortable working with other staff - doctors and nurse managers, whereas the other half finds it comfortable working together. Fifty-nine (85.5%) nurses find it uncom-fortable working in the presence of their head nurse around them and 49 (70%) finds it uncomfortable working in the pre-sence of doctors.</p>\r\n	<p>\r\n		<strong>Relationship of stress with study variables (work stressors)</strong></p>\r\n	<p>\r\n		There is a significant difference between all the study variables with stress (Table 2). Findings indicated a significant difference between nurses with post basic and nurses without post basic training (p&lt;0.05). Thus, it indicates that nurses working in ICU without post basic training find intensive care nursing stressful. Nurses with less years of working experience, experienced more stress compared to nurses with many years of working experience (p=0.0001). A significant difference between work envi-ronment, nurse patient ratio and nursing critically ill patients and stress was iden-tified among the nurses here. This indica-tes that nurses who work in a high tech environment(p=0.0011), a high nurse pa-tient ratio(p=0.0321), working relationship with staff/professionals (p=0.0021) and nur-sing critically ill patients (p=0.0011) are the main cause of stress for nurses working in ICU.</p>\r\n	<p>\r\n		<strong>Coping mechanisms</strong></p>\r\n	<p>\r\n		The five coping mechanisms adopted by ICU nurses were identified. Prayers (100%) and relaxation (100%) methods were the common methods used in coping stress. Physical exercises were only used by some (35.7%) of the nurses whereas more than half (60%) of the nurses relieved stress by ventilating their feelings and sharing their problems with others. Some (35.7%) nurses were found to relieve their stress by taking rest in between work.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		Findings of this study indicated that the prevalence of stress among nurses in ICU, HUKM is high, as symptoms of stress were experienced by 100% of the nurses. Three categories of stress symptoms investigated in this study comprising of physical, psy-chological and changes in behaviour re-vealed interesting findings. The common physical stress symptoms experienced by nurses in ICU were headache, being easily susceptible to viral infections, back pain, insomnia and weight loss. The psycho-logical stress symptoms experienced by many of the nurses is high which consisted of fatigue, anxiety, boredom, poor concen-tration at work, irritability, loss of interest in work and depression. However, the type of changes in behaviour experienced was committing errors at work and having frequent conflict with colleagues and other staff. The result was consistent with the study findings of Milazzo (1988) and Yip (2001). Stress at work did not indulge any of the ICU nurses to be involved in drugs and alcohol as strong spirituality and reli-gious background may have prevented the nurses from doing so.</p>\r\n	<p>\r\n		Knowledge was found to have influenced the level of stress among ICU nurses as the majority (82.5%) of the nurses in this study did not have a post basic course but were posted to ICU to work. Lack of knowledge among ICU nurses was also identified as a main indicator of stress among ICU nurses (Steffen 1980). Find-ings also indicated that the majority of the nurses in this study experienced stress when nursing critically ill patients. This si-tuation was unavoidable as most patients in ICU were either critically ill or required resuscitation. A post basic course in ICU is necessary for nurses working in a special-ized unit like the ICU as it demands a high level of knowledge and skill in handling cri-tically ill patients (Kelly &amp; Cross 1985; Sawatzk 1996)</p>\r\n	<p>\r\n		The significant association between work stress with less experience concurs with previous findings (Huckabay &amp; Jagia 1979; Oisen 1977; Stone et al. 1984). Huckabay &amp; Jagia (1979) again investigated further the association between work stress and less working experience and concluded that &ldquo;once the nurse has the knowledge and skill required, the degree of stress is reduced. However, Sawatzk (1996) con-cluded that nurses perceived inadequate knowledge as challenging. Pagana (1990) also reported that nursing students&rsquo; initial clinical experiences were perceived as sig-nificantly more challenging than threat-ening.</p>\r\n	<p>\r\n		Nurses working in ICU are more often confronted with life sustaining treatment decisions and ethical issues concerning issues of patient care management. Death and dying are frequently encountered and this poses a high level of stress for the nurses working in ICU. This finding is con-sistent with the findings of Sawatzk (1996) and Bartz &amp; Maloney (1986). Study findings among ICU nurses in Hong Kong also reported the causes of stress being nursing critically ill patients, shortage of nurses, increasing workload and interper-sonal relationship among the staff (Lau &amp; Chan, 1995).</p>\r\n	<p>\r\n		Nurses were found to have more than one patient under their care. Nurses find it very stressful when nursing two to three patients under their care. According to Coghlan (1984) shortage of nurses is the main factor causing nurses to care for two to three patients. The inability to meet the needs of patients is affected by the nurse&rsquo;s workload and ultimately patients, and the nurses suffer. Nursing stress ultimately can have an impact on the patient and com-promise patient care (Gentry et al.1972; Rich &amp; Rich 1987). However, according to Payne (2001), despite heavy workload being a frequently reported stressor, it is suggested that some stress may be necessary for optimum functioning.&nbsp; But, of course, heavy workload may lead to other negative consequences.</p>\r\n	<p>\r\n		Maintaining a professional relationship among working colleagues is important as it results in a harmonious working environ-ment. Teamwork and collaboration need to be strong or else nurses will find difficulties in speaking about problems. They will not be able to resolve conflicts in a healthy manner and also may not be able to par-ticipate in clinical decisions. This is agreed by Cottrell (2000) that poor professional relationship may cause a stressful working environment. Therefore, specialized know-ledge and competency are required in or-der for nurses to communicate and colla-borate effectively which will thus improve patient care. In fact, both physicians and nurses find difficulty when confronted with inter-professional problem due to lack of ability and unwillingness to accept res-ponsibility (Maxfield 2005).</p>\r\n	<p>\r\n		An unsafe environment may also cause a stressful environment for nurses. This find-ing is consistent with the findings of Briner (1999). The ICU work environment is phy-sically, cognitively and emotionally deman-ding. Stressful experiences may lead to work-related problems such as staff con-flicts, absenteeism, lowered morale de-creased productivity and ultimately burnout (Stechmiller &amp; Yarandi 1993; Bailey 1980).</p>\r\n	<p>\r\n		Stress can be alleviated by the avail-ability of coping resources such as social support. Nurses in this study were found to have utilized quite effective coping strate-gies such as prayers and relaxation. How-ever, nurses should be taught the appro-priate coping skills. A stress inoculation training could be implemented to prevent total burnout among ICU nurses (Payne 2001). Several studies, however, have identified &ldquo;Painful problem solving&rdquo; as the most frequent coping strategy being used by nurses in other countries (Payne 2001, Healy 2000; O&rsquo;Brien &amp; Delongis 1996).</p>\r\n	<p>\r\n		<strong>RECOMMENDATIONS AND CONCLUSION</strong></p>\r\n	<p>\r\n		This study has important implications for critical care nursing practice and administration. Critical care nurses work in an environment that is highly stressful due to the increased responsibility in the management of patient care. ICU nurses are confronted not only by the increasing demand for quality care by patient&rsquo;s family and the organization, but also by the demand for technological excellence. The origins of stress among ICU nurses in HUKM has now been identified and because stress has an impact on the health and well-being of critical care nurses, it is therefore important for organi-zations to take measures to relieve stress among ICU nurses. Recommendations like encouraging ICU nurses to take up post basic ICU courses and increase staff quota can help reduce stress levels among ICU nurses as nursing is regarded as a very potentially stressful occupation.</p>\r\n</div>\r\n', 1),
(401, 398, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Night eating syndrome (NES) is a syndrome characterized by morning ano-rexia, evening hyperphagia and insomnia. The nocturnal food ingestion is 50% &gt; of daily calories intake and it occurs three times a week for a period of 3 months. NES is commonly associated with obesity and often has an underlying psychosocial problem (Allison et al, 2005). Though it is a fairly common syndrome it is often being overlooked. More often than not the emphasis is more towards medical and cosmetic reasons without paying serious attention on the underlying psychosocial issues which often lead to an unsuccessful outcome.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	Mr AW, A 65 year old Malay widower, a retired army sergeant, was referred to the psychiatric clinic for psychiatric assess-ment. He had multiple medical illnesses and was chronically feeling depressed and had problems with excessive weight gain since the age of 41 (for 24 years). During the same period he retired from the army and said that his wife left him to elope with her ex-boyfriend. The symptoms worsened when his son who was staying with him decided to move out in 2002. He became more depressed and had insomnia, re-maining awake at night and could only sleep by 0500 hours. When he could not sleep he would cook despite not feeling hungry. He ate a lot of high calory and junk food such as fried rice and chicken, nug-gets, cream, biscuits, cake or ice-cream. He also had large amounts of food stocked up in the refrigerator which he kept in his bedroom. He had peculiar eating patterns when he would skip breakfast and take a late lunch. His urge to eat increased during the night. However, he had no compensa-tory behaviour such as self-induced vomit-ting, consume laxatives, pills or diuretics. He was diagnosed with diabetes, hyperten-sion, ischemic heart disease and conges-tive cardiac failure for 10 years. With these illnesses he was admitted several times. Currently, his physical state and illnesses are stable and are under control. In the past he was admitted to the psychiatric ward for depression but was discharged well without medication. There was no history of manic swings. His father was a traditional healer and an alcohol depend-ent; he was rather passive and was ineffec-tive as a father. His mother was severely obese and was described as very strict, harsh and domineering. All his siblings were obese. At school, the patient was an average student and joined the army at the age of 21. He was promoted to sergeant four years later until he retired at the age of 41. Subsequently he worked as a security guard for 20 years until the age of 60. He is now survived on his pension. His perso-nality traits are a mixture of obsessional and borderline traits, characterized by punctuality, determination, impulsiveness, low self-esteem and chronic boredom. His weight was 120kg, height 167.5 cm with waist circumference of 159.5 cm. His body mass index (BMI) was 45.</p>\r\n<p>\r\n	The management consists of a multi-disciplinary teams comprising of a dietician, psychiatrist, psychologist, physician and surgeon. The patient was put on Escitalo-pram 10mg to treat his depression and at the same time underwent cognitive beha-vior therapy. The principal of management in this patient is that while the physician takes care of his physical illnesses, the therapist guides the patient to exercise self-monitoring and to keep a food diary. This is a crucial component of the therapy to enhance awareness of his eating habits and pattern. Since his BMI was 45, and with the presence of co-morbid conditions, he was also referred to a surgeon who recommended him to undergo bariatric sur-gery. However, surgery was later cancel-led because the patient could not afford it. The only option left was the non-surgical interventions. The dietician educated him on proper nutrition to help him identify his main source of calories as this would help him to lose and maintain his weight. The therapist also stressed on healthy lifestyle and regular exercises. With the help of therapists Mr AW identified many types of triggers that precipitated his eating epi-sodes which include long gaps between meals and negative emotions such as boredom, frustration and familial stressors. He was then educated on stimulus control techniques, trained in problem solving skill, cognitive restructuring and relapse prevent-ion. He soon responded to treatment and his eating patterns improved and he could sleep well at night. The NES behaviour has stopped and his weight has reduced from 123 to 91kg over a period of 18 months.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The comprehensive management of this case need a multidisciplinary team ap-proach comprising of the assessment and the management of issues related to medical, nutritional, psychological and psy-chiatry problems (Labib 1994). Research has demonstrated that depression, inter-personal problems, boredom, prolonged fasting could all lead to the development of night eating syndrome (Allison et al, 2005). All these are present in this patient. Al-though eating disorders usually develop in adolescence or young adulthood, they could also affect people above 40 and in children. This patient came late for treat-ment which is 20 years later. This is not un-common because usually people with an eating disorder do not reveal their problems or even conceal them due to denial or lack of awareness. They may also feel ashamed or are ignorant of the treatment available (Annie 1999).</p>\r\n<p>\r\n	&nbsp;&nbsp; Although patients with an eating disorder may be unwilling to enter treatment the clinicians could persuade them by showing lots of empathy, understanding and take a non judgmental approach. Of course, the patient&rsquo;s motivation is the key component for success in any kind of treatment. Generally, the psychiatric assessment of patients with eating disorders would center around establishing a correct diagnosis, identifying co-morbidity and evaluating the underlying psychosocial issues and con-flicts (Labib 1994). Obviously the psycho-social issues in this patient are loneliness, boredom and frustration. He also has low self esteem and depression. Many obese individuals would perceive food as an attempt to decrease emotional upheavals.</p>\r\n<p>\r\n	&nbsp;&nbsp; Biologically, a neurobiological investiga-tion showed that there was an association between psychopathology, weight regula-tion and eating behavior (Hasler et al 2004). While dopamine modulates the rewarding effects of food and the develop-ment of obesity (Rye and Jankovic 2002), it also has an important role in the patho-physiology of sleep-wake disturbances (O&rsquo;Reardon et al. 2004). The Serotonin has been implicated in the control of impulses of eating behavior and depression (Ricca et al. 2002). In our case, escitalopram was chosen because it is the best selective serotonin re-uptake inhibitor for those with depression and poor impulse control asso-ciated with multiple medical illnesses for it has less drug interaction property (Sproule et al. 1997). The other important substance is the appetite-regulating hormone. A study reported that ghrelin levels were lower in NES than in control subjects in the early morning hours when patients remained awake (Wang et al. 2001). There was evi-dence to show that psychopathology and weight problems might share common genetic factors. Findings of a twin study demonstrated an association between de-pression and obesity in which familial / genetic factors influence the vulnerability to depression as well as obesity (Hasler et al. 2004). Cultural factors also contributed to obesity. It was noted that people originating from Eastern Europe and living in the United States had a higher incidence of obesity than people from Western Europe (Bray 1976). Generally the contributing factors to the development of obesity are biological factors, abundance of food, decrease physical activity, basic food habits which include food of high calories and emotional disturbances. Regardless of the cause or factors that contribute to obesity, it can create difficulties that will affect emotional well-being, sexuality, physical and occupa-tional identities as well as the social role. Therefore this problem needs an urgent and comprehensive intervention compri-sing of the bio-psycho-social approaches.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	An integrated approach is required to understand the aetiology and treatment of obesity. The successful management of morbid obesity requires a coordination of care from multiple disciplinary groups namely physician, psychologist, nutritionist, surgeon and psychiatrist, with varied ex-pertise working together as a team to assist patient care.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors would like to thank the patient and all staffs who have helped in the management of this case.</p>\r\n', 1),
(402, 399, '<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	<strong>Case 1</strong></p>\r\n<p>\r\n	A 38-year-old woman was diagnosed with thalassaemia major for the past 26 years. She underwent splenectomy 12 years ago and was recently diagnosed to have Hepatitis C. She presented with a two weeks history of progressive painful right neck swelling which was associated with dysphagia, odynophagia and fever. She was referred to our hospital for unresolved neck swelling after a course of oral anti-biotics. On admission, the patient appeared ill, dehydrated, with pallor, jaundice and fever. Examination showed the patient to be hypotensive, associate with tachycardia. There was a diffuse swelling at the right lateral side of the neck measuring about 10x12 cm. The swelling was soft in consis-tency, inflamed and tender. She had trimus, and her oral hygiene was poor. Oropharyngeal examination showed me-dialization of the right lateral pharyngeal wall. Further examination by flexible naso-pharyngolaryngoscopy noted inflamed and edematous hypopharynx. Her full blood count showed a Hb of 5.7g/dL, TWBC of 47.8 x 10<sup>3</sup>/L and platelet count of 267 x 10<sup>3</sup>/L. Her total bilirubin was elevated (188umol/L). Her renal profiles and chest x-ray (CXR) were within the normal range. She was transfused with four pints packed cells and her Hb rose to 9.8g/dL. Broad-spectrum intravenous (IV) antibiotic was started empirically and septic shock was treated accordingly. An urgent computed tomography (CT scan) was performed and revealed a large collection of pus extending from the inferior part of the right parotid gland to the base of the neck (Figure 1a) and medially to the right carotid space at the level oropharynx. The right sternoclei-domastoid muscle was displaced postero-laterally. Multiple subsmall lymph nodes from level I to III were present the largest measuring about 6mm at the level of the submental lymph node. There was also thrombosis of right internal jugular vein (Figure 1b). The patient was further sta-bilized by the hematology team. Right neck&nbsp; &nbsp;exploration&nbsp;&nbsp; and &nbsp;&nbsp;drainage &nbsp;&nbsp;of&nbsp; &nbsp;the abs-cess was carried out under general anes-thesia after the patient&rsquo;s general condition was stabilized. Intra-operative findings re-vealed multiple loculated areas in the right parapharyngeal space, filled with 50cc thick pus with minimal slough. She deve-loped acute pulmonary edema post-operatively and was nursed in the High Dependency Unit (HDU), under close monitoring by the hematology and anesthe-siology team. The pulmonary edema re-solved by day four post operatively. She was then discharged from HDU to the normal ward. The pus and blood culture and sensitivity revealed Klebsiella spp. and both were sensitive to ceftriaxone sodium (Rocephin). She was then put on IV Rocephin 1gm daily with dressing twice daily over the cervical wound. On the tenth post operative day, the patient was discharged and advised to come for daily dressing to the clinic nearest her home.</p>\r\n<p>\r\n	<strong>Case 2</strong></p>\r\n<p>\r\n	A 28 year-old woman, who was diagnosed to have Insulin Dependent Diabetes mellitus (IDDM) for 6 years, was recently diagnosed with Systemic lupus erythema-tosus (SLE) a month ago in which she was started on prednisolone. However, two weeks later she presented with swelling in the right neck. The swelling was associated with dysphagia, odynophagia and fever.&nbsp; However, there was no shortness of breath or stridor. She gave a history of a right retropharyngeal abscess four years prior to the present condition during which drainage was performed. Clinically she was febrile, with a temperature of 39<sup>0</sup>C on ad-mission. Neck examination revealed a ten-der swollen ill-defined area involving the superior part of anterior triangle of the right neck. The skin overlying it was inflammed and tender. There was trismus and the oral cavity showed bulging of the right pharynx-geal wall with pus noted. She was started on intravenous amoxicillin-clavulanic acid and metronidazole. Contrast enhance CT scan of the neck demonstrated multilo-culated, parapharyngeal and retropharyn-geal abscesses of the right neck with cervical lymphadenopathies (Figures 2a and 2b). She underwent incision and drainage on the second day of admission. Culture studies revealed Klebsiella spp. sensitive to amoxicillin-clavulanic acid. Microbiological culture for tuberculosis was negative. The patient made a rapid recovery and was discharged home after six&nbsp;days.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The&nbsp; parapharyngeal&nbsp; space occupies an in inverted pyramidal area bounded by multiple components of the fascial system. The inferior limitation of this space is the greater cornua of the hyoid bone. The superior margin of the space is the skull base. Its medial border is formed by the lateral pharyngeal wall (ie, the superior pharyngeal constrictor and the tonsillar fossa). The posterior border is formed by the prevertebral fascia. The lateral border is the ramus of the mandible and the deep lobe of the parotid (Amar et al 2004). The anterior boundary is the pterygomandibular raphe. The parapharyngeal space can be subdivided into two compartments by the fascia of the tensor veli palatine muscle which runs obliquely through the parapharyngeal space from the eustachian tube and styloid process to its insertion on the lateral pterygoid plate (Myers et al 1997).</p>\r\n<p>\r\n	The internal maxillary artery, inferior alveolar nerve, lingual nerve, and auriculo-temporal nerve comprise the pre-styloid compartment. Infections in this compartment often give significant trismus. The post-styloid compartment contains the carotid sheath (carotid artery, internal jugular vein, vagus nerve) glossopharyn-geal, hypoglossal nerves, sympathetic chain and lymphatics. It also contains the accessory nerve, which is somewhat protected from the pathologic processes in this region by its position behind the sternocleidomastoid muscle.</p>\r\n<p>\r\n	An odontogenic infection can cause a parapharyngeal abscess. It usually spread contiguously from the mandible or maxilla into the sublingual, submandibular or masticatory spaces and then spread into the parapharyngeal space directly. The most common symptoms of a parapharyn-geal abscess are neck pain, odynophagia, dysphagia,neck swelling, fever, and trimus. Lateral pharyngeal wall medializa-tion is the most common presenting sign in parapharyngeal abscesses (Bottin et al 2003). Both the patients presented with progressive painful neck swellings, asso-ciated with odynophagia, dysphagia and trimus.</p>\r\n<p>\r\n	Diabetes Mellitus (DM) is the most common systemic disease associated with parapharyngeal abscesses (Sethi et al 1991, Huang et al 2004, Bottin et al 2005). It has been established that lipopolysa-charide by bacteria and infection, mediate up regulation of cytokines synthesis. As shown in case 2, she was diagnosed to have IDDM six years ago, and she also gave a history of the retropharyngeal abs-cess, which was treated surgically. Her immune-compromised state further deterio-rated after she was treated with predniso-lone for her Systemic Lupus Erythematous (SLE). Chronic hepatitis was also found one of the commonly associated with para-pharyngeal abscess (Bottin et al 2005). Our first patient has thalassaemia major, underwent splenectomy and just recently diagnosed tobe a hepatitis C carrier. This makes the patient more susceptible to infection.</p>\r\n<p>\r\n	Laboratory investigations usually reveal-ed an elevated white blood count as in our patients, except for patients with HIV infection or AIDS (Shashinder et al 2007). Bacteriologic pattern of parapharyngeal abscess is usually a mixed growth, which includes anaerobes and aerobes. Strepto-coccus viridians were the most commonly isolated organisms in the non-diabetic patients, consistently high with rate of odontogenic infections. Klebsilla pneumo-niae was the most common organisms in diabetic patients (Huang et al 2005). In our patients, Klebsiella spp. was cultured. Broad-spectrum intravenous antibiotics should be started as soon as the diagnosis is suspected, because of the wide spec-trum nature of the involved microor-ganisms. The chosen antibiotics should cover the suspected bacteria and later can be modified according to clinical response based on bacteriological culture and sensitivity results. Discussion with the microbiologists is important to decide the choice of antibiotics, if the patients do not respond to the initial antibiotic.</p>\r\n<p>\r\n	Contrast-enhanced CT scans (CECT) is the gold standard in the evaluation of deep neck infections. CT scans provides valuable information for the site and the extent of infection. Abscesses were seen as low-density lesions with rim enhance-ment, occasional air fluid levels, and locu-lations (Bottin et al 2003). We considered CECT to be part of the routine invest-tigation. CT scan of the chest may be helpful if extension of abscess into the mediastinum was suspected. Otopanto-gram provide useful information in para-pharyngeal abscess of odontogenic origin (Bottin et al 2003).</p>\r\n<p>\r\n	The conventional method of surgical approach for the parapharyngeal space incision and drainage is through an external skin incision made about two finger breath below the mandible. The sternocleidomastoid muscle and great vessels was retracted posteriorly. The parapharyngeal space was entered just anterior to the posterior belly of the digas-tric and under the submandibular gland. Blunt dissection is used to break up any loculations (Amar et al 2004). The wound is left opened for continued drainage and dressing. We used this method in mana-ging our patients in view of better pus collection and ease of dressing.</p>\r\n<p>\r\n	Another method is via the intraoral approach. This procedure is performed under general anaesthesia. The Boyle&rsquo;s mouth gag is placed in the patient&rsquo;s mouth. The mouth is opened to obtain adequate exposure of the oropharynx. The lateral pharyngeal wall is palpated intraorally, or sometimes bimanually to localize the abscess. Once localized, an 18-gauge needle is inserted transorally through the lateral pharyngeal wall and into the abscess cavity. The then pus is aspirated and incision made in the overlying mucosa. The whole dilatation with a large clamp is performed in a longitudinal manner. This wound is left opened for several operative days to allow continued drainage of the abscess into the oropharynx (Amar et al 2004). It is a safe and effective procedure, provided the CT scan obtained preope-ratively shows the abscess is located medial to the great vessels of the neck (Amar et al 2004).</p>\r\n<p>\r\n	Duque reported another surgical intervention whereby intraoperative ultra-sound is used during any external ap-proach to the parapharyngeal space abs-cess when the abscess is difficult to identify with standard anatomic dissection (Duque et al 2007).</p>\r\n<p>\r\n	Feeding should be addressed in view of prolonged course of the disease, which needs nutritional support by nasogastric tube feeding. Tracheostomy may be an option in cases of difficult intubations or incidental rupture of the abscess (Alaani et al 2003). Both of our patients were nursed in HDU for closed monitoring of the general condition. In case 1, she developed acute pulmonary edema. She had aggressive treatment in HDU and was transferred to medical ward on day 4 post operation. Delay in treatment of parapharyngeal abscesses can result in life threatening complications such as airway obstruction, mediastenitis, vascular complication, septi-cemia and mortality.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Even though the advent of the antibiotic has dramatically decreased the incidence of deep neck infection, they remain pre-valent especially in immune-compromised patient. Early and aggressive management of parapharyngeal abscess can prevent major complications such as mediastinitis, vascular complications, septicaemia, retro-pharyngeal abscess mortality. Contrast en-hanced CT scans has preoperative diagnostic value in parapharyngeal abscess and surgical intervention remains the treat-ment of choice.</p>\r\n', 1),
(403, 400, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Pleomorphic adenoma (PA) is characterized by great histological diversity and myoepithelial cells are considered responsible for production of the extracell-ular matrix. It is the most common of all salivary gland neoplasms, accounting for 50-70% of benign parotid tumours. Salivary gland tumors are comparatively rare disorders in the paediatrics population, com-prising of only 1% of all the head and neck tumours and less than 5% of all the salivary gland tumours (Bentz et al. 2000). We report an 8-year-old female with PA arising from the right submandibular gland.<br />\r\n		&nbsp;</p>\r\n	<p>\r\n		<strong>CASE REPORT</strong></p>\r\n</div>\r\n<div>\r\n	<p>\r\n		An 8-year-old Malay girl presented with a swelling below the right mandible of 10 months duration. The swelling was persis-tent and not associated with pain, fever, loss of weight, loss of appetite or related to meals. There was no history of exposure or contact with tuberculosis.</p>\r\n	<p>\r\n		Physical examination revealed a right submandibular mass measuring 2x2cm. The mass was non-tender, firm on palpation, with normal overlying skin. The mass was mobile and there was no cervical lymph node palpable. The floor of the mouth and Wharton&rsquo;s duct showed no abnormality. Other systemic examinations were unremarkable. Fine needle aspiration revealed cellular smears composed of a mixture of epithelial and stromal compo-nents. The epithelial cells were arranged in loose sheets as well as singly distributed displaying plasmacytoid nuclei with bland nuclear chromatin and dense cytoplasm. The stromal component is composed of fibrillary chondromyxoid substance admix-ed with spindle-shaped mesenchymal cells (Figure 1). These cytological features are consistent with PA. Under general anes-thesia, submandibulectomy was carried out. Intraoperative examination revealed a tumor measuring about 3x2cm originating from the inferior portion of the submandi-bular salivary gland. The superior portion of the gland was free of tumor. The tumour was easily separated from the adjacent tissue. &nbsp;</p>\r\n	<p>\r\n		The histopathological examination show-ed a well-circumscribed and encapsulated mass composed of an admixture of epithet-lial, myoepithelial and stromal components. Marked keratinisation and squamous cell metaplasia were present (Figure 2). There were no necrosis, nuclear atypia, hyaline-sation, invasion of adjacent tissue or abnormal mitotic activity noted. Salivary acini were present at the surrounding area. The operative and histological findings indicated that the tumor was completely re-sected and was benign.</p>\r\n	<div>\r\n		<p>\r\n			Postoperative recovery was uneventful. The patient is currently free from disease 24 months after surgery.</p>\r\n		<p>\r\n			<strong>DISCUSSION</strong></p>\r\n		<p>\r\n			Salivary gland neoplasms are rare in children. Not all masses arising in a salivary gland represent tumours of glandular origin. The most common non- epithelial salivary gland tumors are heman-giomas (59.2%) follow by lymphangioma (27.5%) (Bentz et al. 2000). 85% of benign epithelial salivary gland tumours arise from the parotid gland and only 10% in the submandibular gland (Rapidis et al. 2004).</p>\r\n		<p>\r\n			PA is the most common benign type and is found with high incidence in the subman-dibular glands, while muco-epidermoid carcinoma is the most common malignant epithelial salivary gland tumour found in children (Cruz Perez et al. 2004). Most of these tumours occur in the second decade of life (Cruz Perez et al. 2004). This finding differ with Bentz&rsquo;s study (Bentz et al. 2000), which showed a mean age at presentation of 7.2 + 0.7 years. Pleomorphic adenoma has a female predilection with a female : male ratio of 4.4:1, while malignant tumours affected both genders similarly (Cruz Perez et al. 2004).</p>\r\n		<p>\r\n			While relatively rare, PA of the subman-dibular gland presents a therapeutic chal-lenge in the paediatric population. PA typically present as a slow growing and painless mass of salivary glands, with predilection for recurrence and risk of malignant transformation (Rodriguez et al. 2007). The incidence of malignancy frequently showed a correlation between the length of the history of the PA and the development of carcinoma. It has been reported that the risk of developing malignancy is about 1.5% up to 5 years and increases to 9.5% after more than 15 years (Seifert et al. 1991).</p>\r\n		<p>\r\n			Besides the parotid and submandibular gland, PA also occurs at the palate (Lopez-Cedrun et al. 1996), trachea (Baghai-Wadji et al. 1006)and the lacrimal gland (Faktorovich et al. 1996).</p>\r\n		<p>\r\n			Fine needle aspiration (FNA) biopsy establishes the diagnosis with good accu-racy when sufficient specimen is obtained and a skilled pathologist is available (Rodriguez et al. 2004). The positive predictive value of FNA biopsy for PA was 99 % (Tew et al. 1997). However, in children, FNA often require sedation, and there is always the risk of insufficient material for diagnosis. Repeated biopsies or biopsies under computed tomography (CT scan) guidance may be necessary to establish the histological diagnosis. How-ever, some paediatric patients like in this case can tolerate FNA without local anesthesia.</p>\r\n		<p>\r\n			Ultrasonography is a well-tolerated me-thod of evaluating salivary gland masses in children. The technique does not require sedation and does not expose the patient to irradiation. Boccato et al. (1998) advocated that ultrasound should be the first modality in the investigation of salivary gland masses in children, in view of differ-rentiation between intra-glandular and extra-glandular lesions. They also conclu-ded that vascular lesions could be seen clearly with the use of Doppler ultrasound.</p>\r\n		<p>\r\n			CT scan and magnetic resonance ima-ging (MRI) complement clinical evaluation.&nbsp; CT scan requires shorter imaging time and provides excellent spatial resolution, even though it has limited soft tissue charac-terisation. MRI provides excellent soft tissue characterisation and has no expo-sure to irradiation, however, it is costly, is more time consuming and is more likely to require sedation in young children. Both modalities are useful in distinguishing bet-ween solid and cystic masses and differ-rentiating lymph nodes from salivary gland tumours (Rodriguez et al. 2004). Imaging characteristics of a PA on MRI have typically been described as a well-circum-scribed tumour that is hypointense or isointense to muscle on T1 and hyper-intense on T2 (Rodriguez et al. 2004). An enhancing capsule on T1 imaging, with contrast and lobulation of the tumour are also predictive of PA (Ikeda et al. 1996).</p>\r\n		<p>\r\n			Surgical excision is the most common diagnostic measure and is often thera-peutic. Primary surgery determines the success or failure of the operation for PA of the submandibular gland, because every recurrence increases the risk for further recurrences (Rodriguez et al. 2004). The first operation should involve extirpating the entire gland which could minimize the risk of recurrence. Complications included tumour recurrence, transient and perma-nent marginal mandibular nerve weakness, ranula formation, postoperative fluid collec-tion, and cellulitis (Hockstein et al. 2004). Untreated PA has a 2 to 25% risk of malignant change (Rodriguez et al. 2004).</p>\r\n		<p>\r\n			<strong>CONCLUSION</strong></p>\r\n		<p>\r\n			A high index of suspicion is required for non-inflammatory lesions in the submandi-bular region to avoid undue delay in definitive treatment.Pleomorphic adenoma does occur in children although rare. Properative diagnosis with FNAC has proven to be highly accurate. We advo-cate surgical excision because of the young age group and the chances of malignant transformation later in life.</p>\r\n	</div>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(404, 401, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION</strong></p>\r\n	<p>\r\n		Stroke is a major public health problem worldwide. It is the third most common cause of death after coronary heart disease and cancer (McKay &amp; Mensah 2004). Looking into worldwide incidence of stroke, Sarti et al. (2000) found that the incidence of stroke only differs slightly between countries, after being standard-ized for age and sex. The incidence rates for subjects between the ages of 45 and 84 were between 300 per 100,000 population and 500 per 100,000 population annually. In Malaysia, epidemiological data from the Ministry of Health in 2002 showed that stroke was one of the top five leading causes of death in our country, with mortality rate of 11.9 per 100,000 population (MOH 2002).</p>\r\n	<p>\r\n		Overall, the incidence of stroke, especially in developed countries, has shown a declining pattern which largely appears to be due to control of modifiable blood pressure, improved dietary habits and reduced levels of smoking (Hankey 2002). However, the absolute numbers of strokes will continue to escalate in the future due to one single factor, the increasing life span of the world population. This trend is worrying as a significant number of survivors will still be below the retirement age and therefore have many responsibilities involving families and the workforce (Royal College of Physician 2005). Given that three out of four people survive beyond the acute phase, these individuals may lead a longer life with impairment and disability as a cones-quence of stroke. It is therefore imperative that stroke clinicians look beyond the acute phase in order to plan and manage the needs of longer-term stroke patients for them to have best quality of life.</p>\r\n	<p>\r\n		<strong>Longer-term care of stroke &ndash; a definition</strong></p>\r\n	<p>\r\n		The Royal College of Physicians (2004) identifies longer-term care as a period of three to six months post stroke and thereafter (Royal College of Physician 2004). In most patients, longer-term care is the phase which starts soon after transfer from hospital to community, conversely others may identify as the completion of rehabilitation program, including outpatient therapy or any other therapies that has been completed. However, amongst stroke care providers&nbsp; longer-term management is frequently described as &lsquo;a year or more after stroke&rsquo;, given the fact that the majority of prospective cohort studies measuring prevalence of stroke had used one year after stroke as a determinant for longer-term survival (Dennis et al. 1993; Grevenson et al. 1991; Patel et al. 2006).&nbsp;Above all, longer-term care for stroke patients is primarily about managing the consequences of stroke in the best way possible, as it relates to patients&rsquo; quality of life and their relationship with their carers and families, and how best to re-integrate patients into the community with the&nbsp; available resources. In recent years there has been agreement that in overall stroke management, the major elements of longer-term care comprise further rehabilitation, secondary prevention, and involvement in social function (Wilkinson et al. 1997; Murray et al. 2003).This review will address the aspect of further rehabilitation in longer-term management of stroke, as this area of management forms the integral part of overall management for stroke patients that have been discharged home to the community.</p>\r\n	<p>\r\n		<strong>Longer-term consequences of stroke</strong></p>\r\n	<p>\r\n		Stroke is a major health problem in the community, with more and more people surviving and living longer after stroke. However, only a small number of community-based studies have looked into the long-term outcome of stroke patients and very few have assessed functional status beyond a year of stroke onset.&nbsp; Studies that observed survival rates after stroke, agreed that almost half of the patients survived one year or longer, and that the survival rate remained constant up to five years post stroke (Dennis et al. 1993; Grevenson et al. 1991). Disability remains prevalent up to three years post-stroke, with 23-30% having moderate or severe disability (Barthel Index &lt; 15 /20). Patel et al. reported that one in two stroke survivors will remain inactive (Frenchay Activities Index less than 15) at one year after stroke, and a further 30% will require residential care after the first year of stroke (Patel et al. 2006). Wilkinson et al. reported that 46% of stroke survivors needed help with at least one component of activities of daily living (ADL) and named spouse, children and other family members as their main carers (Wilkinson et al. 1997). These two studies used different outcomes in assessing health-related quality of life, but both concurred that the domains that were significantly affected by disability were&nbsp; psychological well being (anxious/ depressed), physical functioning (ADL), physical mobility, general health and social functioning.</p>\r\n	<p>\r\n		Murray et al (2003) and McKevitt et al. (2004), in their reviews of qualitative interviews with stroke patients living more than one year in the community, further explored the nature of problems faced by longer-term stroke patients. Both studies suggested that problems at this stage of stroke were diverse and complex and may change over time. Both reports have identified five common domains that were prevalent at this stage of stroke recovery which include health and social services, problems in accessing information, transfer of care and psychological adjustments. However, these studies were small-scale and geographically localized, focussing more on depth rather than breadth of the problems, and therefore may be only applicable to the selected populations.&nbsp;&nbsp; In summary, there is continuing disability for more than a year after stroke. However the provision of therapy-based rehabilitation services is low. At present there is no agreed consensus about the benefits of providing a service for more than one year after stroke. Documented benefits from community intervention studies are rare in this patient group, as the majority of trials investigated earlier phases of stroke recovery.</p>\r\n	<p>\r\n		<strong>Further rehabilitation :The need for a&nbsp; paradigm shift.</strong></p>\r\n	<p>\r\n		The World Health Organization describes rehabilitation as &lsquo;<em>the combined and coordinated use of medical, social, educational and vocational measures for training and retraining the individual to the highest level of functional ability</em>&rsquo; (WHO 1998). Stroke rehabilitation aims directly and indirectly to increase independence and ability. Not only does it concern prevention of complication, but rehabi-litation encompasses all aspects of the patient&rsquo;s life: physical and psychological health and integration into the community. The stroke rehabilitation approach involves multi-disciplinary team, comprising the patient, the family, therapists, nurses, social workers and physicians. As is evident from overall stroke care, rehabilitation starts as early as hours after stroke and continues up after discharge and into longer term care which involves in-hospital settings to various settings in the community (Hankey 2002). Stroke rehabilitation is a continuous process starting as assessment in the stroke unit and ending only when it no longer produces any positive effect on the patient (Mant et al. 2004).</p>\r\n	<p>\r\n		It was initially thought that progress of stroke patients reached a functional plateau such that little or no recovery occurred after a certain period of time and hence the fixing of three to six months for formal rehabilitation programmes (Pollack &amp; Disler 2002). However, current evidence has demonstrated that stroke survivors showed late functional improvement, even after several years post stroke, and those with continuing decline might be reversed by further rehabilitation input (Mant et al. 2004). Therefore it is now suggested that any patient who has had a stroke reporting significant disability at six months post-stroke should be reassessed and offered targeted re-habilitation to improve functional capabilities (Department of Health 2001).&nbsp;</p>\r\n	<p>\r\n		This emerging evidence in longer-term rehabilitation has brought about a new direction on how rehabilitation should be carried out at this stage after the stroke. It is now suggested that further rehabilitation should move away from the &lsquo;activity&rsquo; phase and address the issue of &lsquo;participation&rsquo; as defined by Wade and Jong as in Table 1 (Wade &amp; Jong 2000). The overall design of this table shows that the approach of rehabilitation towards stroke patients now encompassed a more holistic approach in managing patients; whereby rehabilitation now not only involves intervention to the disabilities and handicaps but helping the stroke patient to improve his/her functional status and to&nbsp; re-integrate into the society. This is particularly important, as patients need to learn to be more independent and try to re-integrate into the community, rehabilitation should concentrate more on social and leisure activities that help improve patients&rsquo; post-stroke functioning level rather than repetitive functional exercises that are commonly performed at present. Several trials have addressed the issue of leisure and social intervention in stroke patients residing in the community, but to this date these studies were small, so that no significant conclusion should be made although individual results showed promising outcome (Drummond &amp; Walker 1995; Logan et al. 1997)).</p>\r\n	<p>\r\n		Trials looking at rehabilitation intervention at this phase of stroke can be divided into those concerned with transfer from hospital to the community and those that were performed in the community itself. The Outpatient Service Trialist systematic review, which looked into 14 community rehabilitation intervention trials involving 1617 patients within a year of having stroke, demonstrated reduced risks of deterioration in ability to the undertake activities of daily living (ADL) and significantly improved the performance of ADL (Outpatients Service Trialist 2002). The meta-analysis also found that common features shared by these studies were that staffs carrying out the intervention were knowledgeable and had specific interest in stroke care and that outcomes were achieved by altering task-associated behaviour. The exact nature and content of intervention were not known, as the studies were heterogeneous in their approach, but it can be concluded that community rehabilitation intervention is both feasible and effective.</p>\r\n	<p>\r\n		On the other hand, there have been a few studies that have addressed the needs of stroke patients more than a year after having a stroke. Two mobility intervention studies demonstrated a short-term improvement in treatment effects as measured in Barthel scores, but these effects were small and intervention was conducted in small scale studies with short intervention periods (Wade et al. 1991; Green et al. 2002). Studies that looked into leisure intervention showed similar trends (Mulders et al. 1989; Werner &amp; Kessler 1996). A Cochrane systematic review is currently in progress in order to establish the evidence of the effectiveness of rehabilitation services more than a year after a stroke, hoping to provide a foundation for better services in the future (Aziz et al. 2006). Without doubt, the element of further rehabilitation is proven to be an important aspect in longer-term care of stroke patients. Consequently there is a need to look again at this aspect of management of the longer-term stroke patients in the community.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		Management of longer-term stroke patients consists of inter-related key elements that are both complex and long-standing. Awareness of the benefits of combining these elements of care to provide holistic management of stroke patients in the community. Current guidelines on stroke have set a clear and explicit standard of care for the components of acute management, secondary prevention and an early rehabilitation programme for stroke patients. Nevertheless, with the emergence of evidence on longer-term stroke management especially in the area of further rehabilitation, a new approach to the rehabilitation of stroke patients in our community needs to be considered. Perhaps what is required now is a new outlook on the longer-term care of stroke patients in Malaysia, which involves the intertwining elements of care and evidence based medicine described above, which will fit into our unique social and cultural&nbsp; way of life.</p>\r\n</div>\r\n', 1),
(405, 402, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	In the competitive health care arena of today, patients&rsquo; satisfaction with health care they received becomes a priority issue. Satisfied patients are more likely to seek and comply with prescribed treatment regimes (Taylor &amp; Benger 2004). Patients who seek emergency care form a diverse group as they have a wide spectrum of health care needs; some people seek care for episodic, acute problems, while others seek care for acute exacerbations of chronic illnesses (Elisabeth 2004; Bruce et al. 1998). The health care provider in the ED is responsible for meeting the various needs of these patients. Treatment plans varies according to the acuteness and problems presented by the patients. Health care providers in the ED need to have an adequate blend of medical and nursing knowledge (Davis &amp; Bush 2003). Adequate medical knowledge is required to identify the correct diagnosis and to prescribe appropriate medical intervention. Educating patients about their condition and treat-ment, addressing effective means of self care and adaptation to change in lifestyle, and if necessary, discussing ways to prevent recurrence of sickness are integral parts of the plan of care.</p>\r\n<p>\r\n	A hospital ED is recognized as the front door where a significant number of in-patient admission take place. According to Gertdz &amp; Bucknall (2001), all patient visits to an ED should begin with triage. In ED, the health provider plays an important role as a gatekeeper toward delivery of care and patient satisfaction (Raper et al. 1999). The tremendous increase in the number of patients visiting ED has contributed to patient dissatisfaction. Satisfaction with the health providers is a predictor of overall patient satisfaction (Elder et al. 2004).&nbsp; Although definitions of patient satisfaction differ slightly from each other, there is general consensus among researchers that patient expectation; demographic charac-teristic and nature of illness are important factors, which contribute to the total level of patient satisfaction (Han et al. 2003).&nbsp;</p>\r\n<p>\r\n	All patients presented to ED HUKM will undergo a two-tier triage process com-prising of primary triage and secondary triage. The 5-level triage category system adopted and practiced is similar to that used in United Kingdom and Australia. A conceptual framework of the ED HUKM is shown in Figure 1. Patients are divided into 5-level triage categories, triage 1 where immediate resuscitation and five beds are available in the resuscitation bays. Emer-gency cases are triaged as triage 2A and 2B. Intervention will be provided in triage 2A less than 10 minutes and six beds are catered to meet patients&rsquo; needs, however, for triage 2B cases treatment is within 30 minutes and there are five beds. Urgent cases are categorized as triage 3. Treat-ments will be rendered not later than 60 minutes. It consisted of five cubicles with five beds and one Plaster of Paris (POP) room available for the patients at ED. Non-urgent cases such as minor conditions or old injuries, awaiting diagnostic tests and cases to be reviewed are placed at triage 4. Patients will be seen by doctor within two hours. Patients are reminded to call health care providers if the need arises, for example, when they feel intolerable pain or have sudden deterioration of conditions which may be life threatening.</p>\r\n<p>\r\n	The aim of the study was to identify factors that influence patient satisfaction with the ED HUKM. From some researcher&rsquo;s personal encounters using observation and interview they have discovered numerous grievances and dissatisfaction expressed by the patients and relatives who have seek services in the ED HUKM.&nbsp; It is hoped that the findings will be able to assist health providers in defining their roles and ultimately to improve the quality of care delivered to emergency patients.&nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This study was undertaken at ED HUKM from January 2007 till March 2007. A convenient sample was drawn from the population of patients attending the ED HUKM over a period of three months. Patients were included&nbsp; if they were at least 18 years of age, able to communicate with the interviewers in Malay or English language and assigned as category triage 3 or triage 4 (HUKM guidelines). Category triage 3 patients are semi-urgent patients who must be seen in the ED and should be re-evaluated during the waiting period. Triage 4 patients are non-urgent patients who may safely wait for a long period to be seen or may be triaged for care to a clinic or follow up. Exclusion criteria were: patients who were in police custody or under protective custody, had history of mental illness, abortion or sexual assault and direct admission to resuscitation room.&nbsp;</p>\r\n<p>\r\n	Patients&rsquo; satisfaction was measured using the Consumer Emergency Care Satisfaction Scale (CECSS) which was developed by Davis in 1988 and revised in 1997 to examine patient satisfaction to triage nursing care (Davis et al. 2003). Questions were translated by our resident Consultant Anaesthetist at ED HUKM to cater for Malaysian patients&rsquo; needs. The CECSS consists of 19 items scored on a 5 point Likert type scale ranging from completely agree to completely disagree. Two subscales comprise the CECSS which is caring (12 items) and teaching (3 items). Four items are negatively worded to minimize response set and are not scored. Each item asks for information about patients&rsquo; satisfaction with ED nursing care.</p>\r\n<p>\r\n	The scoring method established by Davis et al. (2003) had the total possible score ranging from 15 to 75 in which the four negatively worded filter items do not contribute to the total score. A total score of &gt;45 indicates patient satisfaction and scores &lt;44 indicated dissatisfaction. Scores on the caring ranged from 12 to 60 with scores &gt;36 indicating satisfaction and scores on the teaching components ranged from 3 to 15 and a score of &gt;9 indicates satisfaction. Demographic data of the patients include patients&rsquo; age, gender, race, educational level, marital status, employment sector, monthly income, and reasons to seek treatment at ED HUKM.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The majority of participants who reported satisfaction comprised 75 respondents (75%) whereas 25 respondents (25%) were dissatisfied with the triage system used at the ED HUKM. Demographic data such as gender, race, age, educational level, employment sector, monthly income and reasons to seek treatment at ED HUKM are illustrated in Table 1. The results for the CECSS total and subscales are summarized in Table 2, with higher scores indicated greater satisfaction. The t-test was used to examine differences between males and females in terms of patient satisfaction. There were no significant difference (t=0.308, p values &gt;0.05) found between male and female patients with total CESCC scores. A Pearson product moment correlation coefficients was used to examine the relationship between total and subscale patient satisfaction. The results showed positive relationship between total and subscale patient satisfaction scores, caring scores (r=0.905, p value &lt;0.05) and teaching scores (r=0.695, p vales &lt; 0.05).</p>\r\n<p>\r\n	From this study the five items with the highest score ranking were: &ldquo;the nurse/medical assistant explained things in terms I could understand&rdquo;; &ldquo;the nurse/ medical assistant gave me instructions about caring for myself at home&rdquo;; &ldquo;the nurse/medical assistant seemed genuinely concerned about my pain, fear and anxiety&rdquo;; &ldquo;the nurse/medical assistant was skilful in performing his/her duties and the nurse/medical assistant seemed to understand how I felt&rdquo;. Items with lowest scores were as follow: &ldquo;the nurse/medical assistant treated me as a number instead of as a person&rdquo;; &ldquo;the nurse/medical assistant should be more attentive than he/she was&rdquo;; &ldquo;the nurse/medical assistant seemed too busy at the nurses&rsquo; station to spend time talking with me&rdquo;; &ldquo;the nurse/medical assistant understood when listening to my problem and the nurse/medical assistant gave me a chance to ask questions&rdquo;.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The majority of patients were satisfied with caring and teaching aspects rendered by the health providers at ED HUKM. The results have shown that patients had been given clear explanations and adequate details about their treatment.&nbsp; Health care providers played important roles by providing health teaching and advice to their patients whilst they seek treatment at ED HUKM. Satisfied patients are more likely to comply with prescribed treatments and therefore may have better outcome, which bring benefits to both patients and health care providers (Taylor &amp; Benger 2004; Elder et al. 2004). Satisfied patients are more likely to return for follow up treatment. The results indicated that 52% of the patient visit to ED HUKM were referral and followed up cases.</p>\r\n<p>\r\n	Patient satisfaction is not a uniform term as its definition varies. Amongst patients and health care provider researchers, understanding of satisfaction differs. Patient satisfaction can be conceptualized as the degree of congruency between a patient&rsquo;s expectation of care and the actual care received (Elaine et al. 2004; Debenke &amp; Decker 2002).&nbsp; Although definitions of patient satisfaction differ slightly from each other, there is general consensus among researchers that patient expectation; demographic characteristic and patients&rsquo; characteristic are important factors which contribute to the total level of patient satisfaction (Han et al. 2003; Grief 2003). Unlike in this study, the demographic data showed no significant differences with the total scores of patient satisfaction, hence further research need to be done to identify patients&rsquo; characteristic factor.</p>\r\n<p>\r\n	Patients who participated in this study were generally satisfied with the areas covered. The correlations were statistically significant; higher patient satisfaction scores were associated with higher caring and teaching as their contributing factors of the health care provided at ED HUKM. Consequently, there is a growing need for patient education in the delivery of care in order to achieve better health, reduce cost, less complication and revisit to hospital (Faten 2005; Elisabeth 2004). Teaching also assists patient to develop their self care abilities through an increase in knowledge, a more positive attitude and improved skills that enable them to maximize their functioning and quality of life (Davis et al. 2003; Ifantopoulos et al.&nbsp; 1999). According to Elaine et al. (2004),&nbsp; though it is difficult for health care providers to implement health teaching in such an acute setting, providing simple explanation about their illness could allay fear and anxiety.</p>\r\n<p>\r\n	From this study, CECSS questionnaires with regard to the negative items for example: &ldquo;the nurse/medical assistant treated me as a number instead of as a person&rdquo;; &ldquo;the nurse/medical assistant should be more attentive than he/she was&rdquo; and finally the &ldquo;nurse/medical assistant seemed to be busy at the nurses&rsquo; station to spend time talking with me&rdquo; were among the lowest five scored. This could imply that the attention given by the health care providers was adequate and they were attentive to patients&rsquo; needs whilst at ED HUKM. Besides that, the result indicated that patients were satisfied with the amount of time spent with them and the good communication skill. Moreover, the patients considered that the health care providers were sensitive by not labelling them as numbers rather than individuals. By ranking the negative aspect of the health care providers has shown that patient&rsquo;s impression towards health care providers at ED HUKM were warm and receptive towards their needs. Teaching and caring behaviours of health care providers need to be high-lighted at the ED HUKM.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	We conclude that patients seeking treatment at ED HUKM are satisfied with the delivery of care, health-teaching and caring provided by the health care providers. It is important to know which areas of care in the ED need to be adopted to meet the patient&rsquo;s expectations. Health care providers must encourage the evaluation of patient satisfaction and the fulfilment of patient&rsquo;s expectations. Patients&rsquo; satisfaction will remain an important quality outcomes measure of emergency care in a hospital.</p>\r\n', 1),
(406, 403, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Schizophrenia is characterized by profound disruptive psychopathology which involves thought, perception, emotion and behaviour (McKenna et al. 1997).</p>\r\n<p>\r\n	Patients manifest a broad array of cognitive impairments, including executive functions, memory, attention and language capabilities. Studies had shown that quality of life among patients with schizophrenia were mostly lower than that of the general population Lehman et al. (1982), Priebe et al. (2000) and Pinikahana et al. (2002). The importance of quality of life as an indicator of treatment outcome has been well recognized (Malla and Payne 2005). The objective of our study is to investigate the coping styles and the clinical factors in relation to quality of life among patients with Schizophrenia.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Study design</em></p>\r\n<p>\r\n	This is a cross sectional study examining the quality of life of patients with Schizophrenia in relation to coping style, their demographic profile and clinical factors. The study was conducted at the out patient psychiatric clinic at Hospital Universiti Kebangsaan Malaysia (HUKM).</p>\r\n<p>\r\n	Patients with Schizophrenia who attended psychiatric out-patients clinic between period of June 2002 to December 2002 who fulfilled the inclusion criteria were included in this study. The patients were explained about the study and consents were taken from the patients. The clinical assessment was done by the authors. Subjects were considered to be at remission in Schizophrenia if score less than 9 in BPRS. The diagnosis of Schizophrenia was made by the treating psychiatrists using the Clinical Interview Schedule for DSM-IV Diagnosis.</p>\r\n<p>\r\n	Patients were subsequently subjected to the questionnaire on demographic and clinical factors. They were then asked to complete the questionnaire of Medical Outcome Study (MOS) of 36 items Short Form Health Survey (MOH) SF-36 and CISS to assess the quality of life and coping style respectively.</p>\r\n<p>\r\n	<em>Samples</em></p>\r\n<p>\r\n	All patients attending the psychiatric out-patients clinic were registered. The simple randomized of the subsequence 10<sup>th</sup> patient with the diagnosis of schizophrenia were selected for the study.</p>\r\n<p>\r\n	The inclusion criteria were patients of age 18 to 65 years with sufficient command of the Malay or English&nbsp; and no underlying physical illnesswhile the exclusion criteria include those with history of serious brain injury, serious medical illness or&nbsp; substance abuse/ dependent, mental retardation and BPRS score of more than 9.</p>\r\n<p>\r\n	<em>Instruments used</em></p>\r\n<p>\r\n	1.&nbsp;&nbsp;&nbsp;&nbsp; Clinical Interview Schedule for DSM IV Diagnosis.</p>\r\n<p>\r\n	2.&nbsp;&nbsp;&nbsp;&nbsp; Brief Psychiatric Rating Scale (BPRS).</p>\r\n<p>\r\n	3.&nbsp;&nbsp;&nbsp;&nbsp; Medical Outcomes Study (MOS) 36-item Short Form (SF-36).</p>\r\n<p>\r\n	-&nbsp;&nbsp;&nbsp; The MOS-36SF was developed for the Medical Outcomes Study and has been tested and validated extensively. It is a quality of life instrument. A bilingual question-naire, containing the original English Language version and a translated developmental Bahasa Malaysia version was used by Sararaks et al. (2001).</p>\r\n<p>\r\n	4.&nbsp;&nbsp;&nbsp;&nbsp; Coping Inventory for Stressful Situation (CISS).</p>\r\n<p>\r\n	-&nbsp;&nbsp;&nbsp; The Coping Inventory for Stressful Situations (CISS) was developed by Endler and Parker (1990). The Malay version of the CISS has been used in patients who could not understand English. In the local studies Azhar, (2002), Yusni, (2002), Suthahar, (2003), and Emanuel, (2006) had used the same questionnaire in her study on coping style, self-concept, and stress level among the psychiatric nursing staff and the casualty nursing staff.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	The data was analyzed using the Statistical Package for Social Service, SPSS (SPSS Inc, Chicago Illinois, USA, version 13). The relationships between the study para-meters were analyzed using the appropriate statistical tests.</p>\r\n<p>\r\n	Independent t-test and Chi-square are used to determine significant different between two groups. Non-parametric test was done for the data which was not normally distributed i.e. Mann-Whitney U-test, Kruskal-Wallis test and Spearman correlation. The parametric test was use for data that is normally distributed i.e. Independent t-test, Analysis of Variance (ANOVA) and Pearson correlation. Multiple Linear Regression was done for the multivariate analysis with the level of significant of p&lt;0.05.</p>\r\n<p>\r\n	<em>Ethical consideration</em></p>\r\n<p>\r\n	This research project was approved by the Research and Ethical Committee, Faculty of Medicine, Universiti Kebangsaan Malaysia.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 92 patients with schizophrenia from a total of 106 patients who fulfilled the criteria were included in the study. Of those who were eligible for the study, 14 subjects (13.2%) declined to participate. The age range was 19-58 years old. There were 50 Malays, 30 Chinese and 10 Indians. All the details demographic and clinical data were listed in table 1.</p>\r\n<p>\r\n	The quality of life (QOL) of patients with schizophrenia in relation to their demo-graphic and clinical factors were examined. The Exploratory Data Analysis and normality test was done to the quality of life (SF-36) score. For the patient with schizo-phrenia, the score on vitality and physical component summary was normally distri-buted, while the other domains of the quality of life score were not normally distributed.</p>\r\n<p>\r\n	<em>i)&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; </em><em>Quality of life in relation to ethnic.</em></p>\r\n<p>\r\n	There was a significant different in the domains of role emotion, social functioning, mental health and mental component summary of the quality of life among the ethnic groups (p&lt;0.05) (table 2). In term of role emotion and social functioning, Chinese and Malays were better than the Indian. In term of mental health and overall mental health the Chinese was better than others.</p>\r\n<p>\r\n	<em>ii) &nbsp;&nbsp; Coping styles</em></p>\r\n<p>\r\n	The most commonly used coping style among patients with schizophrenia was distraction component of avoidance-oriented coping and the least common was emotion-oriented coping (table 3).</p>\r\n<div>\r\n	<p>\r\n		<em>iii) &nbsp; The relationship between quality of life and coping styles</em></p>\r\n	<p>\r\n		The relationship between the quality of life and coping style among patients with schizophrenia was assessed by using the Pearson correlation for normally distributed data and Spearman correlation for data that was not normally distributed.</p>\r\n	<p>\r\n		There was a positive correlation between the task-oriented coping in relation to the quality of life in term of vitality (r<sub>s</sub>=0.32, p=0.002), general health profile (r<sub>s</sub>=0.31, p=0.002), social functioning (r<sub>s</sub>=0.21, p=0.04), mental health (r<sub>s</sub>=0.31, p=0.003) and mental composite score (r<sub>s</sub>=0.27, p=0.009) (table 4).</p>\r\n	<p>\r\n		This positive correlation coefficient indicates that there is a statistically significant (p&lt;0.05) linear relationship between these variables, the more task-oriented coping the patient used, the better the quality of life in term of vitality, general health profile, social functioning, mental health and also the overall mental health.</p>\r\n	<p>\r\n		There was a negative correlation between the emotion-oriented coping and all the domain of quality of life. This negative correlation coefficient indicates that there is a statistically significant (p&lt;0.05) linear relationship between these variables, the more emotion-oriented coping the patient used, the poor is the quality of life (table 4).</p>\r\n	<p>\r\n		There was also a positive correlation between the avoidance-oriented coping and vitality (r<sub>s</sub>=0.22, p=0.04). This positive correlation indicates that there is a statistically significant (p&lt;0.05) linear relationship between these variables, the more avoidance-oriented coping the patient used, the better the quality of life in term of vitality. But there was no correlation between the distraction component of avoidance-oriented coping and various domain of the quality of life. This is not statistically significant (p&gt; 0.05) and did not show linear relationship between these two variables.</p>\r\n</div>\r\n<p>\r\n	There was significant positive correlation between social diversion component of avoidance oriented-coping with quality of life in term of vitality (r=0.42, p=0.0005), general health profile (r=0.26, p=0.01), mental health (r=0.28, p=0.006) and mental composite score (r=0.30, p=0.004). The more social diversion component of avoidance oriented&ndash;coping the patient used the better in the quality of life in term of vitality, general health profile, mental health and mental composite score (table 4).</p>\r\n<p>\r\n	Multivariate analysis of multiple linear regression showed there were statistically significant between the domains of the quality of life and the clinical factors i.e. ethnic groups, occupational status, atypical antipsychotics and number of admissions (p&lt;0.05) (table 5).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The quality of life among patients with schizophrenia is significantly associated with ethnic, occupational status, atypical antipsychotics and number of admissions. The Chinese has better quality of life in term of overall mental health as compared to other races. This could be explained by the facts that they were mostly on atypical antipsychotics.&nbsp; Studies had showed that atypical antipsychotics were associated with good QOL Karow et al (2002). The Chinese were also had a better economical status and could therefore afford to purchase these costly medications. While those who were employed compared to the unemployed were normally had a better financial standing and could secure the basic physical as well as the psychosocial needs namely food, accommodation and had a higher self-esteem, these would in turn lead to a better QOL.</p>\r\n<p>\r\n	There was a negative correlation between number of admission in relation to the QOL in term of role physical and physical component summary (p&lt;0.05). Those patients who were frequently admitted reported more role limitation because of physical problem. But the number of admission could be an indicator for the severity of illness, those who had more admission could indicate poor compliance, poor social support etc.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp; The quality of life among people with schizophrenia is not significantly associated with age groups, marital status, education level, duration of illness, gender, family history of psychiatric illness and antipsychotic medicines.</p>\r\n<p>\r\n	&nbsp;&nbsp; However in the multivariate analysis of the quality of life in patients with schizophrenia the QOL are statistically associated with ethnic, occupational status, antipsychotic medication (p&lt;0.05) and number of admissions. Those employed and receiving atypical antipsychotic have better quality of life.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp; There was a positive correlation between the task-oriented coping and social diversion component of avoidance-oriented coping in relation to quality of life. On the other hand, there was a negative correlation between the emotion-oriented coping and quality of life.</p>\r\n<p>\r\n	&nbsp;&nbsp; There are several limitations that had been acknowledged in this study. The result of this study should be interpreted with caution.</p>\r\n<p>\r\n	&nbsp;&nbsp; In this study important factors that could influence the result were not considered. Confounders such as adverse life event, support system and adherence to treatment should be controlled Macdonald et al. (1998), Russo et al. (2000) and Ruggeri et al. (2001). This is a hospital-based study and is not a community-based. The samples in our study were patients who were regular with follow-up, had good adherence to medications with a close monitoring by treating specialists and were also in remission state. Therefore the result may not be able to generalize to patients with Schizophrenia as a whole.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	Authors would like to thank all staff and patients who had participated in this study.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(407, 404, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Turner syndrome is one of the most common chromosomal abnormalities affecting 1 in 2500 newborn females (Rosenfeld 1994). It is characterised by short stature, gonadal dysgenesis, con-genital heart disease and renal anomalies. The syndrome is also characterised by a variety of somatic features including neck webbing, cubitus valgus, short neck and widely set nipples. The cytogenetic ab-normality associated with Turner syndrome was first described by Ford and co authors in 1959 (Ford 1959). Since then, a variety of other karyotypic findings associated with Turner syndrome have been determined. The classical 45, X is identified in about half of the patients. The remaining half have either structurally abnormal sex chromosomes (for example 46,X,i(Xq)) or are mosaics with other cell lines with normal (46, XX) or abnormal sex chromo-somes (Jacobs 1997). In addition, a cell line containing the Y chromosome is present in 5% of patients and a further 3% of cases have an unidentifiable marker sex chromosome, presumably derived from a Y chromosome (Magenis 1980).</p>\r\n<p>\r\n	The Y chromosome specific gene SRY is one of the key genes involved in human sex determination. The SRY gene encodes a testis specific transcription factor that plays a key role in sexual differentiation and development in males and is located on the distal region of the short arm of the Y chromosome (Sinclair 1990). SRY expression initiates a network of gene activity that transforms the undifferentiated gonad, genital ridge into testis. Studies have confirmed that Turner syndrome patients with a Y chromosome &ndash; derived material in their genome, may go on to develop gonadoblastoma later in life (Manuel 1976, Gravholt 2000).</p>\r\n<p>\r\n	&nbsp;It has been proposed that all female patients with Turner syndrome with a 45, X karyotype carry a cell line containing two sex chromosomes at a low level of mosaicism (Held 1992). However, the low level of mosaicism is undetectable by standard conventional cytogenetic ana-lysis.&nbsp; Previous studies have indicated that it was important to identify the type and degree of mosaicism since it appears to affect the prognosis and occurrence of stigmata and morbidity (Barrenas 2000, Landin 2001).</p>\r\n<p>\r\n	The laboratory diagnosis of Turner syndrome involves the identification of chromosomal abnormality using genetic methods. Conventional cytogenetics has been the gold standard in the identification of this chromosomal abnormality. Recently, molecular cytogenetic studies such as <em>FISH</em> are rapidly becoming part of clinical practice in diagnosing Turner syndrome and the presence of Y chromosomal material.</p>\r\n<p>\r\n	Conventional cytogenetic analyses are capable of identifying Y chromosomal material in 4%-20% of patients with Turner syndrome (Jacobs 1997). However, Y chromosomal material may be present in only a few cells, and therefore, routine conventional analyses may miss the Y chromosome. The use of molecular techniques in detecting the presence of Y chromosome material is becoming increasingly important in determining those at risk of developing gonadoblastoma.&nbsp; Many studies have reported that molecular studies such as <em>FISH</em> are far superior to conventional cytogenetic analysis in the identification of the Y chromosomal material and the detection of mosaicism (Gravholt 2000, Quilter 1998, Robinson 1995, Hanson 2001, Alvarez 2003).</p>\r\n<p>\r\n	The main aim of this study is to compare the results of conventional (karyotyping) and molecular cytogenetics (<em>FISH</em>), and discuss the advantages and limitations in the diagnosis of Turner syndrome.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A total of eight patients with clinical features of Turner syndrome were seen in the endocrine outpatient clinic in Hospital Universiti Kebangsaan Malaysia (HUKM) between 2004 and 2006. Five millilitres of peripheral blood was drawn from patients and collected in ethylene diamine tetracetic acid (EDTA) tubes. The blood samples were then sent to the Cytogenetics Unit, HUKM for conventional cytogenetic and <em>FISH </em>analyses.</p>\r\n<p>\r\n	<em>Conventional cytogenetic analysis</em></p>\r\n<p>\r\n	Metaphase cells were obtained from PHA stimulated blood lymphocytes following standard protocols. Slides were stained by conventional Giemsa banding method.</p>\r\n<p>\r\n	<em>Fluorescence in situ hybridisation (FISH) analysis</em></p>\r\n<p>\r\n	FISH analysis was performed on the same peripheral blood samples harvested for cytogenetic analysis. Ten micro litres of dual-colour probe cocktail consisting of SRY (SpectrumOrange-labelled, orange) and CEP X (SpectrumGreen-labelled, green) probes (Vysis, USA) was applied to the sample and contained with coverslips sealed with rubber cement. The sample and probe were co-denatured and hybridised using the Vysis HYBrite Denaturation/Hybridisation System. The HYBrite unit was programmed to allow 5 minutes of denaturation at 73&deg;C, followed by overnight hybridisation at 37&deg;C. Post-hybridisation wash was performed in 0.4X SSC/0.1% NP-40 (72&deg;C, 2 minutes) followed by a wash in 2X SSC/0.1% NP-40 (room temperature, 1 minute). The slides were air dried in the dark, then counterstained with 10 micro litres of DAPI (4,6-diamidino-2-phenylindole). The <em>FISH </em>signals were visualised using Vysis filter sets and an Olympus BX51 epi-fluorescence microscope attached to a <em>FISH</em>View image acquisition and analysis system for FISH (Applied Spectral Imaging, Germany).</p>\r\n<p>\r\n	<em>SRY probe</em></p>\r\n<p>\r\n	The SRY gene is located within 10kb of the pseudoautosomal region of Yp.&nbsp; The LSI SRY probe is used for detecting deletions of SRY or presence of the gene in rearrangements involving the X chromo-some, autosomes and marker chromo-somes.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Karyotyping </em></p>\r\n<p>\r\n	Five out of eight cases were found to have a mosaic karyotype with conventional cytogenetic analysis (Table 1).&nbsp; All of the mosaics involve a monosomy 45,X cell line in combination with at least one other cell line;&nbsp; one case with a normal cell line, two cases with a cell line with at least one structurally abnormal X chromosome, and two cases with a cell line containing a possible Y material (Table 1, Figure 1). The remaining three cases were non - mosaics; two cases were 45,X (Figure 2) and one case showed a karyotype of 46,X,i(X)(q10) (Table 1).</p>\r\n<p>\r\n	<em>FISH</em></p>\r\n<p>\r\n	<em>FISH </em>was used to identify the presence of the SRY gene and to determine the origin of the marker chromosome.</p>\r\n<p>\r\n	Two out of eight cases analysed were found to have the SRY gene (Figure 3). One case (TS2) showed presence of Y chromosomal material in 50% of the cells. This confirms the finding of the karyotypic analysis that the marker identified was a Y chromosome.&nbsp; In the second positive case (TS6), 88% of the cells showed Y chromo-somal material. However, it was observed that the SRY gene in this second case (TS6) was translocated onto one of the autosomes (Figure 4). The other six cases were negative for the SRY gene.</p>\r\n<p>\r\n	Analysis with CEP X probe confirmed that all cases were consistent with the karyotypic findings. However, in one non-mosaic case identified by conventional karyotyping, (TS7), the <em>FISH</em> analysis showed an additional normal cell line (Table 1, Figure 5).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Turner syndrome is characterised by a range of clinical stigmata and cytogenetic analysis is the definitive investigation for patients with this syndrome. In Turner syndrome, there is an increased risk of developing gonadal neoplasms if Y chromosome material is present (Gravholt 2000). Therefore, the detection of Y chromosome material in Turner syndrome is of diagnostic importance.</p>\r\n<p>\r\n	Conventional cytogenetic analysis (karyotyping) detects Y chromosome mosaicism in about 5% of patients with Turner syndrome (Alvarez 2003).&nbsp; However, if Y chromosome material is present in only a few cells, it may be missed by the conventional method which routinely analyse 30 metaphases only.</p>\r\n<p>\r\n	&nbsp;Some authors have recommended the PCR method in the investigation of Y chromosome material in all patients with Turner syndrome, or in cases when a marker of undetermined origin is found (Rosenfeld 1994, Iezzoni 1997). Most of these studies had utilised PCR method as a screening procedure in identifying Y chromosome material, and <em>FISH</em> to confirm the findings of the PCR. Although PCR is a highly sensitive method in identifying Y chromosomal fragments, it is difficult to quantify the proportion of Y positive cells using the PCR method. <em>FISH</em> analysis, on the other hand, can easily quantify the proportion of cells containing Y chromo-some material. Studies have indicated that the higher the proportion of cells bearing the Y chromosomal material, the more likely it is for the patient to develop gonado-blastoma (Roc&iacute;o 2005, Prandstraller 1990). Therefore, the risk assessment of deve-loping gonadoblastoma in Turner syn-drome can be made possible by using <em>FISH</em> analysis.</p>\r\n<p>\r\n	<em>&nbsp;FISH</em>can also quantify the level of X chromosomal mosaicism. The purpose of quantifying the level of X chromosomal mosaicism is important because the type and degree of mosaicism appears to affect prognosis, and the occurrence of stigmata and morbidity (Barrenas 2000).</p>\r\n<p>\r\n	In this study, <em>FISH</em> was used to identify and confirm the presence of Y chromo-some material or the SRY gene in patients with Turner syndrome. It was also used to analyse the level of mosaicism identified.&nbsp; The results of <em>FISH</em> were then compared with the karyotypic findings (Table 1).</p>\r\n<p>\r\n	In our study, two (TS2 and TS6, Table 1) of the eight patients with Turner syndrome were found to have the SRY gene by <em>FISH </em>analysis. Of these two, one patient (TS6) was originally thought to have a non mosaic 45, X karyotype by conventional method karyotype. However, <em>FISH</em> analysis confirmed the presence of the SRY gene in her peripheral blood. The <em>FISH</em> analysis also revealed that the SRY gene has trans-located onto one of the autosomes (Figure 4). The findings of this particular case indicated that although conventional cyto-genetic method (karyotyping) is the definitive diagnosis of Turner syndrome, karyotyping alone cannot be used to identify the autosome for which the SRY gene has translocated onto. Further molecular cytogenetic analyses such as spectral karyotyping (SKY) is required to confirm the origin of this autosome. SKY is a molecular cytogenetic technique which combines <em>FISH</em> and direct labelling technology for identification and analyses of complex chromosomal translocations.</p>\r\n<p>\r\n	The Y chromosome has an indistinct banding pattern and it can sometimes be difficult to determine the structure of the Y material from G banded preparations alone. In our study, one patient (TS2) was found to have a mosaic karyotype with a marker chromosome by conventional cyto-genetics. Subsequent <em>FISH</em> analysis con-firmed that the marker chromosome was a Y chromosome bearing an SRY gene. This shows that determination of the origin of the marker chromosome can be made possible with <em>FISH </em>studies.</p>\r\n<p>\r\n	Previous studies have found that the majority of patients with Turner syndrome, who either had a 45,X karyotype or seen to have a Y chromosome or a marker cyto-genetically, tend to be positive for Y chromosome material or SRY gene. The results of our small study have suggested that mosaic patients with cell lines containing two X chromosomes are less likely to be positive for the SRY gene or Y chromosomal material. However, a larger study is required to validate this finding.</p>\r\n<p>\r\n	Several studies have reported a cor-relation between phenotype and genotype in Turner syndrome. In general, 45,X monosomics display more stigmata and higher morbidity than mosaics (Gotzsche 1994, Verp 1987). Furthermore, mosaics are known to have less cardiac anomalies and a lower prevalence of cardiovascular disease and hypertension than mono-somics (Landin 2001). It is therefore essentially important that the degree of mosaicism is accurately analysed in each patient with Turner syndrome.</p>\r\n<p>\r\n	In this study, the identification of mosaic cases was increased to 88% by using <em>FISH</em> compared to 63% by karyotyping only. The increase in the proportion of mosaic cases was attributed to the detection of cells with the SRY gene in patient (TS6) previously karyotyped as 45,X and also to the presence of 45,X cells in patient (TS7) with 46,X,der(X) karyotype. In this study, the mosaicism identified by <em>FISH </em>indicates a more favourable prognosis for these two patients. Therefore, we strongly re-commend <em>FISH </em>as a quick and cost effective tool in diagnosing Turner syn-drome and assessing the degree of mosaicism.</p>\r\n<p>\r\n	The risk of developing gonadoblastoma increases with age in Turner syndrome. In young patients with 45,X/46,XY or 45,X/46,X,+mar(Y), the risk is essentially zero (Verp 1987). The risk is dramatically increased from 15% to 20% by the age of 30 years. However, a more recent data suggested a lower risk of only 7% to 10 % (Gravholt 2000). At the time of analysis, patient TS6 was 32 years old and the age of patient TS7 was unknown. None of our patients positive for the SRY gene or Y chromosome were known to have had gonadectomies or gonadoblastoma. It is essentially important for these patients to be followed up since they are at risk of developing gonadoblastomas.</p>\r\n<p>\r\n	In conclusion, <em>FISH</em> technique is more superior to conventional cytogenetic ana-lysis (karyotyping) in the identification of Y chromosomal material in Turner syndrome. We strongly recommend <em>FISH</em> as a quick and cost effective tool in the diagnosis of Turner syndrome and assessing the level of mosaicism.</p>\r\n', 1),
(408, 405, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Breastfeeding (BF) is widely recognised to be the optimal way to nourish and nurture infants. Even with the addition of comple-mentary foods in the second half of the first year, BF continues to be of nutritional, immunological, and psychological signifycance well into the second year and beyond (Cadwell 2002). The Innocenti Declaration on Protection, Promotion and Support of Breastfeeding 1990 recognised that BF was a unique process with manifold advantages (WHO/UNICEF 1990).</p>\r\n	<p>\r\n		In order to achieve global goal for optimal maternal and child health and nutrition, the Baby-Friendly Hospital Initiative (BFHI) was launched in 1991 by the World Health Organisation (WHO) and United Nations Children&rsquo;s Fund (UNICEF) to ensure that all maternity facilities, whether free standing or in a hospital, became centres of BF support. A maternity facility could be designated &#39;baby-friendly&#39; when it did not accept free or low-cost breast milk substitutes, feeding bottles or teats, and had implemented 10 specific steps to support successful breastfeeding (UNICEF website).</p>\r\n	<p>\r\n		The Ministry of Health of Malaysia had been exemplary in its pursuit of the BFHI accreditation.In March 1998, Malaysia was recognised by WHO as the only third world country, after Sweden and Oman, to have successfully accredited all its government hospitals as Baby-Friendly Hospitals. As of July 2005, 117 hospitals in Malaysia were accredited as Baby-Friendly Hospitals (UNICEF 2005a). Despite all these effort, the State of the World&rsquo;s Children 2005 (UNICEF 2005b) reported that only 29% of babies in Malaysia were exclusively breastfed for the first 6 months from 1996-2005.</p>\r\n	<p>\r\n		<strong>OBJECTIVE</strong></p>\r\n	<p>\r\n		The general objective of this study was to assess the knowledge and attitude towards breastfeeding among our pregnant mothers. The prevalence of those who plan to breastfeed and the main source of information about BF were determined.</p>\r\n	<p>\r\n		<strong>METHODS</strong></p>\r\n	<p>\r\n		This study was conducted in Selayang Hospital, which was a tertiary referral centre with 960 inpatient beds and 20 clinical disciplines located in the Gombak District of Selangor. It was a Baby-Friendly hospital supporting breastfeeding. The Obstetric and Gynaecology Department took over full obstetric and antenatal coverage including networking of the Northern Zone of Selangor. In 2006, the total number of deliveries in the hospital was 10,790 cases.</p>\r\n	<p>\r\n		This was a cross-sectional study using structured questionnaires to interview pregnant mothers attending the antenatal clinic from 1/3/07 to 31/3/07.Universal sampling method was used. All pregnant mothers who were present for the antenatal sessions within the study period were recruited.</p>\r\n</div>\r\n<p>\r\n	Using a structured anonymous question-naire, the sampled pregnant mothers who had given verbal consent to participate in the study were interviewed. The question-naire encompassed demo-graphic data, knowledge and attitude towards breast-feeding, and main source of information.&nbsp; Respondents who answered at least 70% of the questions correctly were perceived as having high level of knowledge. Those who answered at least 70% of the questions correctly regarding attitude were regarded as having positive attitude towards BF.</p>\r\n<p>\r\n	The questionnaires were pre-tested in a different group of antenatal patients prior to the actual study. Analysis of the collected data was done by using SPSS version 12.0.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 288 pregnant mothers attended the antenatal clinics during the study period. Seventy were excluded from the study as 42 of them refused to be interviewed, 27 left the clinic before being interviewed and one of them being a foreigner had language barrier. The remaining 218 were successfully recruited making the response rate of 75.69%, which was within acceptable percentage.</p>\r\n<p>\r\n	The respondents&rsquo; age ranged between 16 &ndash; 46 years (mean age of 30.4 years, SD of 5.3 years). The majority of respondents were multigravida (72.9%), being Malay (61.5%) and were married (99.5%). Most of them were housewives (45.4%), attaining at least secondary educational level (85.3%). Only 29.4% of respondents had total household income of less than RM 2,000 per month. Table 1 shows the demographic data of the respondents.</p>\r\n<p>\r\n	Almost all (96.8%) intended to breast-feed their newborns, the rest were either had no intent or was undecided.</p>\r\n<p>\r\n	Majority of the pregnant mothers (74.8%) scored high marks in breast-feeding knowledge with total score of more than 70%. Most of them (95.9%) knew that breast milk was the best infant food. Almost all of the respondents agreed that breast milk was good for infant&rsquo;s resistance towards disease (96.3%), and BF increased bonding between mother and infant (97.2%). Breast milk was mentioned as easy and economical for their infant by 93.1% of the respondents. Most mothers (82.1%) knew that BF helped them in childbirth recovery. Breast milk was recog-nised as a well-balanced nourishing food for infant, and good for avoiding infant allergy by 97.2% and 83.9% of respon-dents respectively. Mothers knew that breast milk helped infant teeth develop-ment (64.2%) and filled up the stomach more easily (73.9%). Majority of the respondents (71.6%) agreed that BF helped to reduce maternal weight. Many mothers (45.4%) thought they should stop BF when infant and/ or mother was sick. Most of them (63.3%) would give clear fluids to the newborn even in exclusive BF to prevent dehydration. Colostrum was known to be good for their infants by 82.6% of respondents. The majority of mothers (81.2%) knew the correct age of weaning. The responses to knowledge of BF are shown in Table 2.</p>\r\n<p>\r\n	Most mothers (34.9%) gathered BF information from the mass media e.g. television programs, Internet, magazine, newspaper and etc; followed by antenatal class organised by midwives and nurses in the hospitals and health care centres (32.1%). They also gained knowledge from other mothers with BF experience (27.5%) and other personnel (5.5%) e.g. nurses or doctors in clinics or hospitals. Figure 1 summarises the main source of information about BF.</p>\r\n<p>\r\n	When assessing their attitude, 183 (83.9%) mothers responded positively towards BF. Most of them (86.2%) considered BF as being easier than feeding with infant formula. Most mothers (92.2%) denied BF had negative effect on marital relationship. Majority of respon-dents (91.7%) did not think that BF would make it difficult in taking care of their family. Only 56.9% of the mothers believed they could breastfeed their babies with modesty anywhere. The commencement of BF straight after delivery was known to be important by 98.6%. Many respondents (68.3%) agreed with the banning of the usage of bottles and teats in any hospital unless prescribed by doctor. Overall, 84.9% of the respondents believed that the community encouraged BF rather than infant formula feeding. Many of them (83.0%) would not stop BF when there was discouragement from their husband. The responses of attitude toward BF are shown in Table 3.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	This study showed that most mothers were knowledgeable and supportive towards BF. The overall prevalence of those who intended to breastfeed their babies was high at 96.8%. Though the respondents were pregnant mothers who had not started BF, their early decision would pose a positive bearing after childbirth. A study by Hoyer et al had shown that the time of decision to breastfeed was important for the length of BF (Hoyer &amp; Pokorn 1998). In her study, 77.3% of mothers decided to breastfeed before they went into labour. By making an early choice, mothers made better intellectual and physical preparation. Relevant information could be attained and the decision made after talking with family members. In this study, the &lsquo;almost-all&rsquo; prevalence of intention to breastfeed was consistent with the respondents&rsquo; high level of knowledge and positive attitudes towards BF.</p>\r\n<p>\r\n	The antenatal decision of wanting to breastfeed may not reflect the actual prevalence of BF. Even if a mother was to breastfeed, the commitment of whether it was partial or exclusive and the length of BF was yet to be determined. A study in the neighborhood capital of Bangkok showed that 95% of mothers breastfed their infants up to 3 months, but the prevalence of exclusive BF was relatively low at 62.4% (Li et al.1999). We have no local data on the prevalence of BF among post-partum mothers at 3 months, but it was reported that only 29% of babies in Malaysia were exclusively breastfed for the first 6 months (UNICEF 2005b).&nbsp;&nbsp; The theoretical declining prevalence of BF practice from antenatal to third and sixth months post-partum was an area for further research. The initiation and successful practice of BF may be affected by various factors including the social, psychological, environmental and practical barrier. Each country with its own policy, race, culture and religion could have different perception on infant feeding. In Northern Ireland, the main barriers to BF were incompatible social norms, making it difficult for mothers to breastfeed successfully (Stewart-Knox et al. 2003).</p>\r\n<p>\r\n	Out of 218 respondents, 163 (74.8%) were knowledgeable about breastfeeding with total score of more than 70%. They knew that colostrum and breast milk was the best food, good for resistance against disease and allergy, filling up stomach easily, helpful in teeth development and maternal recovery after birth, increased bonding, was easy and economical. A study in Vietnam showed that mothers were less aware of the advantages of BF in helping the mother to recover from childbirth and that it helped infant&rsquo;s teeth development (Li et al. 2002).</p>\r\n<p>\r\n	In this study the respondents had two main wrong perception i.e. discontinuation of BF when baby and/or mother was sick and clear fluid was unnecessarily administrated in exclusively breast-fed infants. These misconceptions could be influenced by social and cultural beliefs. Public education or breastfeeding cam-paigns should therefore address these misconceived issues appropriately.</p>\r\n<p>\r\n	A high percentage of respondents (83.90%) showed positive attitude towards BF i.e. being easier than infant formula, had no negative effect on marital relationship or family care, would com-mence breastfeeding straight after delivery, agreeable to the banning of bottles and teats in hospital and they would not stop breastfeeding even if the husband discou-raged them. This was an encouraging finding among urban mothers-to-be. A study in Baghdad among medical profes-sionals showed that only 76.8% of general practitioners, 70.1% of resident doctors and 68.8% of medical students had a positive attitude towards BF (Al-Nassaj et al 2004).</p>\r\n<p>\r\n	Although the attitude towards breast-feeding was generally positive, only 56.9% of respondent thought it was appropriate for a mother to breastfeed her baby with modesty anywhere. This was probably due to our social environment, cultural and religious sensitivity. The respondents may believe that self-embarrassment, inade-quate public facilities in providing privacy while BF and difficulty when mothers were working outside home, were barriers towards BF anywhere. This practical issue should be addressed when promoting BF.&nbsp; Antenatal education about the practice of breastfeeding, milk expression and storage may encourage mothers to continue BF anywhere even in the work place.</p>\r\n<p>\r\n	This study showed the strength of mass media in disseminating information in urban society. About one third of mothers (34.9%) gathered BF information from television programs, magazine, newspaper and Internet establishing that BF infor-mation could be effective via public education through the mass media. Another one third of the respondents stated the main source of BF information was from antenatal class organised by midwives and nurses in the hospitals and health care centres (32.1%). Comparing to the Bangkok study, there was much room for improvement as 73.3% of the mothers had already taken courses on breast-feeding and childcare prenatal or post-partum (Li et al.1999). Both antenatal and postnatal education program were influential in achieving successful BF (Hoyer &amp; Horvet 2000).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Most mothers were knowledgeable and had positive attitudes towards BF. The misconception and negative attitudes had been identified and should be addressed appropriately by the NGO and Ministry of Health. Mass media and antenatal class were the main source of information and should be used to promote BF.</p>\r\n', 1),
(409, 406, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Development of new technologies and increase in the number of industries is the scenario observed in most developing countries. More occupations are created alongside these industries and high economic growth is anticipated. In the wake of the creation of new occupations in these industries, highly repetitive motion patterns in the work situation are common. This work situation which involves repe-titive movement and overuse of certain musculoskeletal system is often the cause of pain in the upper limb region. This can give rise to Work Related Upper Muscular Disorders (WRULD) (Maeda et al 1982). WRULD is also synonymous with work related musculoskeletal disorder (WMSD), cumulative trauma disorders (CTD), repetitive strain injury (RSI), occupational overuse syndrome (OOS) and occupational cervico-brachial disease (OCD) (Yassi 1997).</p>\r\n<p>\r\n	&nbsp;&nbsp; WRULD is defined as any changes around the tendon-muscle unit, peripheral nerves and vascular system of the muscle (Grieco et al 1998, Hagberg 1996, Armstrong et al 1993). WRULD can be divided into Type 1 and 2, where Type 1 is denoted by specific clinical symptoms and can be grouped into specific clinical diagnosis such as lateral epicondilitis, De Quervain disease and Carpal Tunnel syndrome. The latter type of WRULD is classified as Type 2 and it is a non-specific condition affecting certain parts of the upper limb from neck to fingers, which commonly presenteds with pain (Hutson 1997).</p>\r\n<p>\r\n	&nbsp;&nbsp; The incidence of WRULD is high among industrial workers and computer user office workers (Hocking 1987). The USA Labor Statistics Unit (1994) reported a 32% ab-senteeism due to WRULD as a result of repetitive motion and overuse condition. These disorders involved the wrist joint (55%), shoulder (7 %) and back (6 %). In Australia, there were 343 cases diagnosed with WRULD among 1000 telephone operator, between 1981 till 1985 (Hocking 1987).</p>\r\n<p>\r\n	&nbsp;&nbsp; The risk factors for WRULD are multi factorial and work has been recognized as one of the risk factor (Grieco et al 1998, Hagberg 1996, Armstrong et al 1993). These work conditions include repetitive motion, use of force, ergonomic problem related to inappropriate positioning, expo-sure to vibration, cold temperature and psychosocial environment at the work place (Williams and Westmorland 1994, Stobbe 1996, Hagberg 1996, Colombini 1998).</p>\r\n<p>\r\n	WRULD has been reported in many occupations such as cashiers, meat cutters, packing operators, telephone operators and musician. In Malaysia, few studies on WRULD have been done. Cardosa and Wan Yusof (1992) found that 50% of data processor personnel in a public department and 66% of tele-communication operators had experienced neck and shoulder pain. Abd Ghani (1995) reported that 50% out of 1670 workers from six factories complained of upper limb and back pain. Studies on WRULD among telecommunication workers in Malaysia have been limited, except for a study by Premalatha in 1995, where it was found that prevalence of WRULD among 323 telecommunication workers was 31.2%. With the passing of years and increase use of new technology which concentrates on creation of new machines with better accessibility, the prevalence of WRULD may have significantly changed since the last study in 1995 (Premalatha 1995). For comparison, a current study on tele-communication workers was carried out. Hence, with this aim in mind, a study was done to determine the prevalence and risk factors of WRULD among tele-communication operators.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This cross sectional study was carried out among female telephone operators working in a telecommunication centre under the Directory Unit in Kuala Lumpur between December 2000 and May 2001. There were 79 female workers included in the study and they make up the entire force of the Directory Unit. Permission to carry out the study was obtained from the Medical and Ethics Research Committee of Uni-versiti Kebangsaan Malaysia.</p>\r\n<p>\r\n	&nbsp;&nbsp; The female workers were interviewed by the researcher using questionnaire inquiring information pertaining to socio demographic factors such as weight, height, age, ethnicity, menstrual cycle, hormone replacement therapy (HRT), over-time work, duration of employment in the present unit and in the telecommunication centre; occupational history, past medical history and symptoms and signs of neck and upper limb disorders. The physical examination included inspection of the neck and upper limbs to confirm any disorders or previous injury. The power and tendon reflexes of the biceps, triceps and brachioradialis were also tested. The sensation for both pain and light touch of the upper limbs were tested using a needle and cotton wool. A full neurological examination was performed in the upper limbs. Additional signs looked for included the Phalen&rsquo;s test, Tinel&rsquo;s test, Fikelstein sign, muscle atrophy and range of movements at the shoulder, elbow and wrist joints. The height and weight were measured using measuring tape and Salter bathroom scale. The bathroom scale was calibrated after each weight measurement.</p>\r\n<p>\r\n	&nbsp;&nbsp; The telecommunication centre operates from 7 am to 7 pm everyday and staff may either have normal or shift working hours. A normal working hour was from 8.30 am to 5 pm (Mondays to Fridays) and from 8.30 am to 12.45 pm on Saturdays. The shift working hours were either 6.5 or 8 hours. The shift work hours is shown in Table 1.</p>\r\n<p>\r\n	Those shift workers were given two days off after 12 working days. The workers had 20 minutes of break time in the morning and one hour of break time for lunch (on Friday two hours lunch break except those working on shift). All activities were fully computerized since 1988.</p>\r\n<p>\r\n	&nbsp;&nbsp; WRULD is defined as the presence of pain in any part of upper limb area that is either continuous or episodic for a day or more&nbsp; or any abnormalities found during&nbsp; physical examination such as muscle atrophy, muscle spasm, pain or tenderness during palpation, reduce range of movement, reduced power or the presence of neurological signs. The Body Mass Index (BMI) was considered normal if the value was between 18 and 25, low if less than 18 and overweight if more than 25. The exclusion criteria included history of injury to the upper limbs including fracture or dislocation which could cause weakness or alteration sensation in the upper limbs, history of neurological disorders such as poliomyelitis, any connective tissue disorders such as SLE and rheumatoid arthritis, endocrinological disorders that could cause carpal tunnel syndrome, or muscle weakness such as thyrotoxicosis and acromegaly and those who had a part time job that increased the risk for WRULD.&nbsp; WRULD was graded using the Japanese grading as explained in Table 2 (NOHSC, 1986).</p>\r\n<p>\r\n	Data collected were later analyzed using the SPSS 9.0 software. The Chi square test was used for categorical data and independent t-test was used for numerical data. The level of significance was set at p&lt; 0.05.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Seventy nine female workers agreed to participate in this study. The response rate obtained was 96.2% (n=75). Three workers were excluded from the study because they had rheumatoid arthritis, history of trauma with neurological signs.</p>\r\n<p>\r\n	&nbsp;&nbsp; The majority of the workers were Malay (92%, n=69) and less than 40 years of age (68%, n=51).&nbsp; Eighty four percent (n=63) were taller than 150 cm, 92% (n=69) were right handed and 50.7% (n=38) were overweight and obese. Most of them had been working for less than 10 years (66.7%, n=50); 53.3% (n=40) worked in shifts of 6.5 hours and the majority (53.3%, n=40) had resting times of 50 minutes per working time. Seven subjects were preg-nant and one respondent was on HRT after total hysterectomy and bilateral oophorec-tomy (Table 3).</p>\r\n<p>\r\n	Thirty six subjects were diagnosed as having WRULD which makes the prevalence of WRULD to be 48% (Table 4). The subjects diagnosed with WRULD were further classified according to five different stages according to severity. The majority of the subjects with WRULD were categorized in to Stage 1 (38.9%, n=14) and Stage 3 (38.4%, n=14). There were 19.4% (n=7) subjects in Stage 2, none in Stage&nbsp;&nbsp; 4 &nbsp;&nbsp;and&nbsp; 2.8% &nbsp;&nbsp;(n=1) &nbsp;&nbsp;respondents classified in Stage 5 (Table 5). None of the recognized risk factors were significant (<em>p</em>&gt; 0.05) in this study (Table 6). Mean duration of employment among subjects with and without WRULD was 11.59 &plusmn; 9.09 and 9.89 &plusmn; 8.48 years respectively. Mean duration of service at the Directory Unit for subjects with WRULD was 8.59 &plusmn; 6.67 years and for those without WRULD, the mean duration of service was 8.14 &plusmn; 5.80 years. None of the employment duration factors showed any association with the presence of WRULD among the subjects (Table 7).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The study was carried out among telephone operators in a telecommuni-cation centre in Kuala Lumpur. Since the workforce consisted of mostly females, male operators were excluded from the study. Furthermore, there are studies quoting females at higher risk to develop WRULD when compared to males (Feuerstein &amp; Harrington 2006). In this study, prevalence of WRULD was 48%. A previous study by Premlatha (1995) has also shown that female telecommunication workers have a prevalence of WRULD of 41.8%. The high prevalence of WRULD in females may be due to ergonomic factors at the work station which were originally designed based on male anthropometric data (Ulin 1993). Other explanation for the high prevalence of WRULD was that women complained of pain more frequently when compared to men (Armstrong 1993).</p>\r\n<p>\r\n	&nbsp;&nbsp; The majority of subjects were Malay, thus, the effect of ethnicity on WRULD is neutralized. Findings of this study did not prove the presence of an association between known risk factors such as BMI and age with WRULD. &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp; Contrary to the findings of this study, Bernard and Fine (1997) showed that age was associated with WRULD. This was due to the combination of degenerative diseases as age increases or natural physiological changes that occurred in tissues whereby any injury occurring will have more significant effect among the elderly compared to the younger people. This may be explained by the presence of survival bias or healthy worker effect whereby workers with health problems will leave the job earlier and those who are well will remain. Ohlsson et al. (1998) inter-viewed 76 ex-assembly operators who claimed that health condition was the reason for them to leave their job.</p>\r\n<p>\r\n	&nbsp;&nbsp; Factors such as overtime work, duration of employment in the present unit and in the telecommunication centre showed no association with WRULD. This could be due to the fact that sample size of the study might be small. Ching et al. (1993) found that there was no association between duration of employment and WRULD among fish processing industries but there was a significant dose-response relationship between the magnitude of risk factors and WRULD for those exposed to the risk factors. Fredrikson et al. (1999) carried out a cohort study and found out that there was an association between overtime work and prevalence of WRULD when age was matched. In this study, the risk factor of overtime work was found not to be significant in contributing towards WRULD since its introduction in the telecommunication centre before this study commenced.</p>\r\n<p>\r\n	Pregnancy and HRT intake have no association with WRULD. Comparisons could not be done due to the small number of pregnant subjects in this study. However, the study by Ching et al. (1993) found that those subjects taking HRT had a twofold risk of developing WRULD when compared to subjects not taking HRT. Considering that the age pool of subjects tended to be from a younger age group, intake of HRT is not common.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	It could be concluded that WRULD is common among female telephone operators in the Directory Unit in the telecommunication centre. Pain and neuro-logical symptoms occurred in almost 40% of the workers. Since none of the risk factors under study were associated with WRULD, an elaborate study with a case-control design is needed in order to match cases and enable researchers to control the effect of confounding factors. This will ensure identification of risk factors pertinent to WRULD in the Malaysian setting. These risk factors will beuseful when developing intervention programmes. Appropriate training and guidelines are also required for easy information dissemination to ensure the safety and health of workers.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(410, 407, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The intensive care unit (ICU) is a highly specialized unit, where medical and nursing staff with specialised skills provides treatment and care to patients with life threatening conditions (Patak et al. 2004). Patient&rsquo;s physiological status is monitored continuously and special procedures and therapies, which cannot be undertaken in the general ward, are carried out in this unit (Stein-Parburg &amp; McKinley 2000). Venti-lated patients who require intensive care unit (ICU) treatment may encounter unpleasant experiences. These expe-riences may include factual incidents and delusional memories of ICU such as dreams, hallucinations and frightening experiencesapart from their ongoingmedical or surgical illness (Patak et al. 2004; Bergborn-Engberg &amp; Haljamae 1998). These experiences have been known to prolong their rehabilitation, leading to poor recovery. Survivors of critical illness from ICU with differing presenting diagnoses have reported phy-sical and psychological problems during their recovery (Jones et al. 2003). Moreover, Rattray et al (2004) &amp; Patak et al (2004), reported that patients who required ICU had untoward experiences lasting well beyond the physical recovery of their illnesses. These incidents may include factual or delusional memories of their ICU stay, and these are usually reported as dreams and hallucinations.</p>\r\n<p>\r\n	The sources of stress on patients in ICU include health care providers themselves, visitors, and environmental factors such as noise, lightning and procedures. Some frequently cited factual memories en-countered in ICU and perceived as stressors to the patients are: tracheal intu-bation, extubation, endotracheal suctioning and physiotherapy. Although, these memo-ries may be worsen by illness, judicious use of sedation can overcome these pro-blems (Roberts et al. 2007).</p>\r\n<p>\r\n	Patients admitted into the ICU can broadly be divided into two types. Some patients are admitted as emergencies such as after a cardiac arrest or major trauma e.g. road traffic accident. On the other hand, admission would be planned for example, after surgery or as a result of an existing medical condition that has deteriorated and needed respiratory support, close monitoring and drug infusions. There may be differences in the level of adaptation between the two types of patients. Pre-admission information, which can be given to patients for &ldquo;elective&rdquo; admission to the ICU, provides a way to achieve better adaptation, while such benefit is not available for an emergency admission (Cornock 1998). For an emer-gency admission, the patient usually expe-riences higher level of stress and anxiety due to a sudden change of environment and unavoidable traumatic encounters (Strahan et al. 2003). It can be judged from a nurse comment as cited by Bergbom-Enberg &amp; Haljamae (1998)<em>&ldquo;even as long as four years after respirator treatment, most patients (90%) who remembered their treatment still recall the situation as unpleasant and stress evoking</em>&rdquo; that patients often regard ICU as a place that is unpleasant and dreadful. According to Stein-Parbury et al (1990) mechanically ventilated patients face many barriers in their communication needs. Inability to communicate with others results in unrecognized pain, feelings of loss of control, anxiety, fear, distress, frustration and depersonalization. Tracheal intubation is often being regarded as the number one problem causing communication difficulties (Roberts et al. 2007).</p>\r\n<p>\r\n	Bergbom-Enberg et al (1998) retro-spectively interviewed 158 patients who were mechanically ventilated from 2 to 48 months and recorded their recall of experiences during their ICU stay. Appro-ximately 50% of the patients were able to remember the experience of being ventilated and most recalled the situation as discomforting, stressful, feelings of anxiety, fear, agony, panic and insecurity. Similarly, Roberts et al (2007) concluded that the inability to speak was identified by all respondents as causing distress such as pain and discomfort from endotracheal tube, suctioning. Other distresses were inability to orientate to time and the intolerable amount of noise. These distressing factors can be aggravated by the health care providers themselves in the delivery of care. Most of the time, the nurses failed to address the patients&rsquo; specific physical and emotional needs because of communication problems (Scragg et al. 2001).</p>\r\n<p>\r\n	According to Roberts et al (2004), over one third of patients have some degree of factual memory related to mechanical ventilation, especially suctioning and removal of the endotracheal tube. Stein et al (2000) stated that factual recall of traumatic incidents e.g. endotracheal tube suctioning may protect patients against anxiety and panic attacks after ICU discharge. Traumatic memories from the ICU such as hallucination may leave patients vulnerable to anxiety, depression, and panic attacks and most feared of all, post traumatic stress disorder. Patients frequently admitted that these memories as extremely vivid and real. Some patients tried to block recall of these traumatic memories and accept them by making them intellectually meaningful (Scragg et al. 2001). Incidental events such as viewing a hospital, future episodes of seeking treatment at hospitals and watching television programs, may cause them to become overwhelmed by the feelings and physiological arousal that can result in depression. Chan (1990) reported similar findings. However, all memories of ICU may not be negative and stressful. Rattray et al (2004) interviewed 109 patients and found 80% of them rating their care as good, over 50% of the respondents reporting that they felt safe and 40% never felt scared while being treated in ICU. Scragg (2001) found that the presence of a caring relationship either by the nurses or family members can have a positive impact and decrease the level of fear whilst in ICU.</p>\r\n<p>\r\n	As most studies in literature series were undertaken in the West, there is limited localdata on patients&rsquo; recollection whilst being mechanically ventilation in ICU. The objective of this study was to investigate recollections of artificially ventilated patients at ICU of HUKM. This study will add on to the body of evidence regarding recollection expressed by mechanically ventilated and the actual level of frustration among our local patients whom by far, are different in demographic and socio-economic entities in comparison to the West.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross sectional study was conducted in ICU of HUKM from January to March 2006. Data collection was done using a structured &ldquo;Intensive Care Experience Questionnaire&rdquo; (ICEQ) adopted and modified from Rattray et al (2004) which consisted of four domains: awareness of surrounding; frightening experiences, recall of experience and satisfaction with nursing care. There were three sections in the data collection form; section A: comprised of 30 questions using Likert scale 0-2 (0-never, 1- some of the time and 2- all the time) to measure what patients felt and remember about their experiences whilst during their ICU stay; section B: 31 questions using Likert scale 0-3 (0 - not applicable, 1 - not stressful, 2 - a little stressful and 3 - very stressful) to measure patients recollection of stress experienced during intubation and on mechanical ventilation in ICU. Section C consisted of demographic data of the patients including patient&rsquo;s age, gender, level of education, marital status, occu-pation, monthly income and length of stay in ICU.</p>\r\n<p>\r\n	<img height="84" src="///C:%5CUsers%5Cpersonal%5CAppData%5CLocal%5CTemp%5Cmsohtmlclip1%5C01%5Cclip_image001.gif" width="544" />Inclusion criteria were: age more than 21 years, ability to orientate to person, place, time and situation at time of data collection, competency and ability to sign an informed consent and requirement of intubation and mechanical ventilation for at least 18 hours with extubation within the previous 72 hours at time of interview. A convenience sample of 45 patients from ICU who met the inclusion criteria were recruited for this study. These time frames were chosen to provide a sufficient amount of time required for the patients to recall their experience while being mechanically ventilated after extubation. Patients were excluded if they had: neurological complications; psychiatric problems, tracheostomy and heamo-dynamic instability at time of data collection. The study was approved by the ethics committee of Universiti Kebangsaan Malaysia, Faculty of Medicine (FF-199-2005) and permission granted from the Director of Hospital Universiti Kebangsaan Malaysia. An illustration of the attributes of this study, as a conceptual framework is shown in figure 1.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Demographic data of respondents are presented in table 1. From the findings of 45 participants, 20 respondents (44%) were aware of their surrounding while 25 respondents (56%) were unaware of their surroundings. However, 31 respondents (69%) reported frightening experiences. In the recall of experience domain, 39 (87%) respondents had actual recall, whereas 6 (13%) respondents were unable to recall their experiences. A high proportion: 43 respondents (96%) reported satisfaction with the delivery of nursing care and only 2 respondents were unsatisfied (figure 2). There was positive correlation between awareness of surrounding and their abilities to recall their experiences (p&lt;0.05). Patients&rsquo; awareness of surrounding shows the strongest statistical significance; a unique contribution to the prediction of their abilities to recall their experiences with beta coefficient value 0.353 and p&lt;0.05.</p>\r\n<div>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		This study revealed that mechanically ventilated patients were aware of their surrounding during ICU stay and able to recall their frightening experiences. They were aware of what was happening to them though they were sedated.These factors are similar to those identified by other studies (Roberts et al., 2007; Scragg et al., 2001). To protect patients from these harmful factual memories during their ICU stay, nurses should be aware of the need to communicate adequately and respect patients&rsquo; rights to dignity and information even though they were on sedation and relaxant. However, Armondo et al (2002) reported that severely ill patients on prolonged mechanical ventilation are likely to be paralyzed and heavily sedated, may suffer from short term loss of memory and may not be fully aware of their ICU surrounding. However, more severely ill patients tend to have poorer recall of their ICU stay.</p>\r\n	<p>\r\n		More importantly, nurses should be more diligent and committed in delivering nursing care to ventilated patients in ICU. Nurses may be able to reduce the stress asso-ciated with endotracheal tube suctioning by providing prior explanations to patients of what they may expect and be gentle during the procedure. They can identify inade-quate levels of analgesia and sedation by assessing patients&rsquo; reactions to the endotracheal tube suctioning. The role of nurses are to help monitor the patients while the anesthetists are responsible for the other management of the patients such as prescribing anxiolytic drugs to reduce patients&rsquo; anxiety, analgesics to reduce pain and also the use of sedatives to increase comfort and improve sleep. A better under-standing of the recollections of traumatic events and stressful experiences reported by patients could allow nurses to better anticipate in patient care and improve their quality of life during ICU stay (Strahan et al. 2003).A large number of patients have predicaments in verifying their perceptions and cope with fears, anxiety and stress through verbal communication (Scragg et al. 2001).</p>\r\n	<p>\r\n		This study adds new information about the relationship of satisfaction between mechanically ventilated patients and the nurses. Our findings further support pre-vious studies by Johnson &amp; Sexton (1990), who stated that, nurse-patient relationship in ICU is deemed to have the power of reducing patients&rsquo; frustration and dissatis-faction. However, when ventilated ICU patients&rsquo; feelings are unmet due to the inability to communicate adequately with nurses, these could aggravate anger, discomfort and anxiety. This will sub-sequently lead to a prolong rehabilitation and poor recovery that exacerbate physical and psychological disability (Rattray et al., 2004).Several studies also revealed similar findings on patient&rsquo;s feelings and experiences related to anger, discomfort and anxiety when they are unable to com-municate effectively (Bergborn-Engberg et al., 1998). According to Roberts et al. (2007), some patients may be unwilling to talk about their bizarre and unreal expe-riences for fear that others may judge them to have mental instability.</p>\r\n</div>\r\n<p>\r\n	It was found in this study that patients&rsquo; awareness of surrounding was the strongest factor statistically and was a unique contribution to the prediction of their abilities to recall their experiences. However, Strahan et al (2003) reported that frightening memories, for example, hallucination or nightmares are potential threats to patients&rsquo; psychological recovery. Patients discharged from ICU needs to have an opportunity to discuss their memories and nightmare as a way to overcome the hurdles to recovery.&nbsp; Consequently, patients should be explored about their unpleasant memories to prevent residual psychological distress, which can result in poor mental health (Jones et al., 2003).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The finding of this study permits nurses to see the problems of mechanically ventilated patients through reports of unpleasant recollections by the patients themselves. Patients reported frustrations in their attempts to make their needs known. Although they are on sedation and relaxant, they are aware of all unpleasant events occurring in the ICU. This infor-mation offers insight into the needs to alleviate patients&rsquo; unpleasant experiences and what the nurses can do to help mechanically ventilated patients.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	I sincerely would like to acknowledge the support of many people especially Asso-ciate Prof.Choy Yin Choy and Dr. Noorfaizan Saaidin for their contribution in this study. My special thanks go to intensive care staff and patients of HUKM for granting permission to conduct this study.</p>\r\n', 1),
(411, 408, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Since time immemorial, the aims of the physician have been to cure sometimes, relieve often, and comfort always. To &ldquo;comfort always&rdquo; and the ability to communicate effectively with patients are fundamental for clinicians (Jason 2000; Ong et al 1995). Evidence-based studies show that doctors&rsquo; interpersonal and communication skills have a significant impact on patient care and correlate with improved health outcomes and health care quality (Rider and Keefer 2006; Nobile &amp; Drotar 2003; Stewart 1995; Stewart et al 1992). Effective communication is needed for doctors to make an accurate diagnosis, to facilitate compliance with treatment by creating a trusting relationship with chronically ill patients and their families, to cope with breaking bad news and dealing with angerand to establish harmonious relationships with colleagues (Royston 1997), support staff and superiors. Dissatisfaction of patients and the public is due more to poor communication than to any other pro-fessional deficiency among doctors (Anon 1993). Communication is a core clinical skill that can be taught and learned (Rider and Keefer 2006) and teaching com-munication skills is now part of the undergraduate medical curriculum in the majority of medical schools world wide (Lloid and Bor 1996).</p>\r\n	<p>\r\n		The Medical Faculty of Universiti Kebangsaan Malaysia (UKM) started to teach communication skills formally from the year 2005 under the new integrated curriculum, consisting of 10 semesters in five years. Before 2005, UKM medical curriculum was more conventional and disciplined-based in which communication skills were not explicitly taught or assessed. The objective of this paper is to determine the perceptions of house officers of UKM from the old curriculum on their communication skills in different aspects. The study findings may also be utilized as base-line data to make comparisons in future research between the products of the new integrated curriculum where communication skills are taught and assessed.</p>\r\n	<p>\r\n		<strong>MATERIALS AND METHODS</strong></p>\r\n	<p>\r\n		A cross sectional study was carried out on house officers at different hospitals in Malaysia who graduated from the UKM in 2004. Data was collected utilizing a standardized questionnaire containing both open and close-ended questions on different attributes of communication skills as competencies. In order to collect the data, questionnaires were mailed through the academic office to the home addresses of all 172 graduates. The questionnaires were posted at around the middle of their housemanship period. Only 32 doctors returned their questionnaires giving a response rate of 18.6%. The data was then compiled and analyzed.</p>\r\n	<p>\r\n		<strong>RESULTS</strong></p>\r\n	<p>\r\n		Table 1 showed the abilities of communi-cation of graduates with different groups of people where 87.5%, 68.8%, 59.4%, 40.6% and 37.5% of the respondents communicated very well with peers, support staff, patients, patients&rsquo; families and their superiors, while 12.5%, 31;2%, 40.6% and 53.1% and 62.5% communi-cated reasonably well with the respective groups of people. Communication abilities of 6.3% of the respondents were not well at all with patients&rsquo; family.</p>\r\n</div>\r\n<div>\r\n	<p>\r\n		Table 2 showed that 21.9%, 50% and 81.2% of the respondents were very well in breaking bad news, counselling patients and taking consent for procedures while 78.1%, 50% and 18.8% were reasonably well in doing so.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		The majority of the house officers claimed to have communicated very well with their peers, support staff and patients. Moreover on comparison, communication with patients was the lowest (59%). Lack of attention was given to families of patients which were reflected by the low (41%) percentage of very good capabilities in communicating with patients&rsquo; families. Only 38% of the respondents claimed to have communicated very well with their supe-riors (Table 1), which seemed to be not enough. Breaking bad news to patients and their relatives is a difficult task for health professionals (Barnett et al 2007). Although it is an inevitable part of medical practice most of us worry about our ability to communicate sensitive and distressing news like loss of well-being, hope, health and relationships (Lloid &amp; Bor 1996). The findings of this study revealed that only 22% of respondents perceived themselves to have very good capabilities in breaking bad news. Competence in patient counselling seemed better (50%) and competence in taking consent for proce-dures was also good (81%) among the respondents (Table 2). Different studies also revealed that the practicing physicians are themselves ill equipped with skills of communication (Zabidi 2000; Waitzkin and Stoekle 1972).</p>\r\n	<p>\r\n		It is now established that patients&rsquo; satis-faction, compliance with medication and disease outcome are all associated with the quality of good communication (Royston 1997; GMC 1993; Simpson, Buckman &amp; Stewart 1991; Ley 1982). Good communication falls into two categories: those of imparting clear information and those of relating empathically with the patient. Good communication is not easy; few can master it without special tuition and constant attention to its effectiveness. Communi-cating effectively involves the core skills of questioning, active listening and facilitating (Lloid &amp; Bor 1996). Competency in communication skills is required at all levels of medical training (Rider and Keefer 2006; IIME 2002; Frank et al 2000; Whelan 1999; Klass et al 1998; LCME 1998). Indeed, the General Medical Council in its recommendations on basic medical edu-cation has rightly stated that &ldquo;on graduation a student should be competent to commu-nicate effectively and sensitively with patients and their relatives&rdquo; (Rees et al. 2002; Royston 1997; Stewart 1984).</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		The majority of UKM house officers&rsquo; of the old conventional curriculum have good communication skills in different aspects. However, the skills of communication with their superiors and the skills of when communicating sensitive and distressing issues like breaking bad news needs to be improved. The low return rate of questionnaires by the house officers and self claim data, with no verification of the claims is the limitations of this study. Further large scale studies are required to reveal more information. Educational managers should emphasize on improving communication skills in all aspects of future doctors during their educational management and planning.</p>\r\n	<p>\r\n		<strong>ACKNOWLEDGEMENTS</strong></p>\r\n	<p>\r\n		The authors would like to express their sincere thank to Prof. Dato&rsquo; Dr. Lokman Saim, Dean and Director of UKM Medical Center for his support to this study.&nbsp; Thanks are also due to the staff of the Academic Office and Medical Education Department of UKM Medical Center through whom data were collected and analyzed.</p>\r\n</div>\r\n', 1),
(412, 409, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		In the coming decade, the global burden of cancer shall change as developing countries adopt a more westernised lifestyle (Parkin 1998). Cancers as a group in Malaysia had risen from 6.7% of all medically certified deaths in 1966 to 10.8% in 1998 (Lim 2002) with the burden of cancer cases approaching the pattern of developed countries.</p>\r\n	<p>\r\n		Based on the National Cancer Registry data, one in four Malaysians will develop cancer during his or her lifetime. The cancer burden was estimated to be of about 40,000 new cases per year per entire population (Looi et al 2004). Furthermore, peculiar to Malaysia, the overall female incidence of 148.4 per 100,000 was higher than the male incidence of 118.9 per 100,000 persons. The ratio of male to female incidence was 1:1.2 unlike other countries such as Singapore (1:0.97), United Kingdom, Norway (1:0.94) and Hong Kong (1:0.76) (NCR 2002).</p>\r\n	<p>\r\n		Cervical carcinoma (CC) was the third most frequently occurring malignancy worldwide. Mortality rates were much lower than incidence rates with an average five year survival of above 50% worldwide. Due to an earlier occurrence, the total years of life lost to the malignancy was substantial (Herrero 1996). In 1990, nearly 80% of CC cases were estimated to occur in developing countries (Parkin et al 1999). CC mortality rates in Malaysia declined from 1985 to 1993 and then increased in 1997. In addition, the incidence of CC was among the highest with an overall incidence higher than all developed countries. The age standardised incidence rate of Chinese in Malaysia was higher than in China, Hong Kong, most parts of Asia, superceded only by some Latin American and African countries (NCR 2002). In 1995, CC was the third most common cause of death due to solid tumours among females followed by carcinoma of the breast and the respiratory tract in Malaysia. In addition, the distribution pattern changed in relation to ethnicity. In Chinese and Malay females, the prevalence of CC ranked second to breast cancer (BC) whereas the pattern was reversed in Indian females (Cheah &amp; Looi 1999).</p>\r\n	<p>\r\n		BC is the most common form of malignancy among women worldwide, accounting for 21% of overall cancer diagnosis. It also caused the most deaths, with almost 50% dying from this disease (Parkin 1998). Despite the fact that Malaysia had good treatment centers for reproductive cancer, mortality rates from BC and CC were high because of the failure of early detection. BC mortality rates have risen since 1985. Between 1993 and 1996, 35% of the BC cases presented at stages 3 or 4, and 93% of these women had tumours with a mean size of 5.3cm (Hisham and Yip 2003). The reported incidence rate of BC in Malaysia appeared lower compared to other developed countries although the difference may be attributable to underreporting which could undermine efforts to acquire accurate figures. Worldwide, a 4-5 fold variation exist with the highest rates observed in Europe and North America and the lowest in Asia (Hisham and Yip 2003; Hisham &amp; Yip 2004).</p>\r\n</div>\r\n<p>\r\n	Women between the ages of 40-59 have a 1 in 11 chance of developing some type of invasive cancers. In comparison, women from ages 1-39 have a 1 in 51 chance of being diagnosed with cancer. The most frequent cancers in Malaysian women were cancers of the breast, cervix, colon, ovary, leukaemias and lung. Together, cancers of the breast, cervix, ovary and corpus uteri, afflicting 30.4%, 12%, 5% and 3.6% respectively made up half&nbsp; (51%) of all cancer cases (NCR 2002). Thus screening especially cancers of reproductive organs as women approached menopause becomes exceedingly important (Givens &amp; Ling 2003).</p>\r\n<p>\r\n	This study reports the incidence of breast and cervical cancers in a general population of urban women aged 45 years who volunteered in a lifestyle intervention program, with hopes of providing a health insight in these women.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Data for this study was obtained from entry assessment of subjects who participated in a lifestyle intervention program. Briefly, healthy women aged 45 years and above with intact uterus, non HRT users were recruited from November 1999 to October 2002 via distribution of flyers to residential areas around the hospital, including subjects from other areas who responded after being informed by friends. After an initial screening of 745 women by tele-phone, 522 subjects were accepted into the study. Five women were later excluded for surgically induced menopause leaving only 517 subjects.</p>\r\n<p>\r\n	Other exclusion criteria included preg-nancy, immigrants, those on treatment for hypertension, diabetes mellitus, hyper-lipidaemia and chronic illnesses. Those with secondary causes of osteoporosis such as a thyroid or endocrine condition e.g. Cushing&rsquo;s disease, hyperthyroidism, hyperparathyroidism, malabsorption, hypo-gonadism and prolonged immobilisation, were also excluded. The study protocol was approved by the hospital ethics committee as part of a Masters of O&amp;G study thesis (Nur Azurah 2003).</p>\r\n<p>\r\n	All subjects gave written informed consent before participation in the study. Recruits were interviewed for medical history and examined by a designated physician who also obtained cervical smears. Height and weight were measured to the nearest 0.1 unit with a balance beam weighing machine and stadiometer (Health-O-Meter, USA). Information acquired by questionnaires included personal characteristics and lifestyle. All activities were carried out in the general gynaecology clinic by trained personnel.</p>\r\n<p>\r\n	Pap smears were taken by the attending physician on the first or second visit. After the first visit, 29 women were disqualified from the study upon discovery of illness needing medication (diabetes mellitus n=9, hypertension n=5, thyroid dysfunction n=1, severe osteoporosis n=1) or surgery (hysterectomy n=5, breast cancer n=6, thymus cancer n=1, nasopharyngeal cancer n=1).&nbsp; Pap smears were not taken for 22 of these women. A mammogram by appointment was done 6-8 weeks after the initial visit in the radiology unit; 24 subjects were lost to follow up. The study reports a total of 495 and 471 subjects for Pap smears and mammogram respectively with subjects followed up to year 2005.</p>\r\n<p>\r\n	Data were analysed using SPSS for Windows version 11.0 software. Continuous variables were expressed as mean and standard deviation, and compared by the Student&rsquo;s t test. Categorical data was compared using the Chi-squared test and relationships with Pearson or Spearman correlation where appropriate, followed by logistic regression analysis. The differences were considered significant if p&lt;0.05.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<strong><em>Subject characteristics</em></strong></p>\r\n<p>\r\n	The average age of participants was 51.27&plusmn;5.35 years old (age range 45-79 years). Over two thirds were Chinese followed by Malays and Indians, reflecting an urban distribution as opposed to the general population with Malays being the major ethnic entity. The study sample comprised of 300 (58.0%) premenopaused (age range 45-58 years) and 217 (42.0%) postmenopaused women (age range 45-79 years), similarly distributed between the major ethnic groups (Table 1). The post-menopausal group at mean age of 55.53&plusmn;5.27 years was 7.4 years older than those premenopausal, 48.14&plusmn;2.67 years (p&lt;0.0005). The mean and median age of menopause was 49.97&plusmn;2.90 years and 50.00 years respectively with no significant difference between the ethnic groups. Malays were the youngest, followed by Chinese, Indians and others with respective mean age of 50.14&plusmn;4.33, 51.51&plusmn;5.53, 54.47&plusmn;7.83 and 57.00&plusmn;10.53, a significant difference (p=0.039). The average and median age of menarche was 13.57&plusmn;1.73 and 13.00 years old. Indians reaches menarche at 12.55&plusmn;1.62 years; earlier than Malays, Chinese and others with a respective mean of 13.36&plusmn;1.32, 13.72&plusmn;1.87 and 13.20&plusmn;0.84 years (p&lt;0.005).</p>\r\n<p>\r\n	The mean height, weight and body mass index (BMI) of the sample population was 1.55&plusmn;0.05m, 59.41&plusmn;9.50kg and 24.64&plusmn; 3.89kg/m<sup>2</sup> respectively. The premeno-pausal group was similar in weight, height and BMI to the postmenopausal group. Malays were the heaviest, 62.39&plusmn;10.00kg with significantly higher BMI, 26.15&plusmn; 4.30kg/m<sup>2</sup>, compared to the others (both p&lt;0.0005). Weight and BMI of Chinese, Indians and other races were 58.30&plusmn; 9.14kg, 58.03&plusmn;7.75kg and 57.20&plusmn;9.76kg; 24.08&plusmn;3.60kg/m<sup>2</sup>, 23.98&plusmn;2.83kg/m<sup>2</sup> and 23.61&plusmn;2.40kg/m<sup>2</sup> respectively. Overall, 56.3% had BMI between 18.5 to &lt;25 kg/m<sup>2</sup>, 32% were overweight and 8.6% obese.</p>\r\n<p>\r\n	<strong><em>Cervical cytology</em></strong></p>\r\n<p>\r\n	Overall, over four fifths of women (83.5%) had normal Pap smear readings (Table 2). Abnormalities in Pap smear cytology were seen in 7.6% comprising 1.3% cervical intra-epithelial neoplasia (CIN), 6.1% HPV infection and 0.2% ASCUS. Yeast infections such as moniliasis and other organisms made up 8.9%.</p>\r\n<p>\r\n	A 2.8 fold higher CIN grade 1 or 2 was demonstrated in postmenopaused women compared to the premenopaused, 2.1% vs. 0.7% although not statistically significant. One premenopausal subject had atypical squamous cell of undetermined signifi-cance (ASCUS) and required colposcopy. In contrast, premenopausal women had a higher infection rate compared to post-menopaused, 11.3% vs. 4.2% respectively (p=0.024).</p>\r\n<p>\r\n	One in seven (6.9%) had unsatisfactory smears, 3.9% versus 10.8% in pre and postmenopaused women respectively. Unsatisfactory smears were 2.8 fold higher among the postmenopaused due to atrophic vaginal changes.</p>\r\n<p>\r\n	The distribution of abnormal cytology between the ethnic groups showed no significant difference. The occurrence of abnormal smears did not correlate with all the variables studied. The study data emphasised vigorous screening for carcinoma of the cervix to be implemented in postmenopausal women as the incidence of invasive CC was higher.</p>\r\n<p>\r\n	<strong><em>Mammogram</em></strong></p>\r\n<p>\r\n	Over 90% of women had normal findings on mammogram while 39 (8.3%) showed some abnormalities (Table 3).In this study, 6 (1.3%) had breast cancer, 17 (3.6%) required a biopsy while 16 (3.4%) needed regular follow up. The distribution of abnormalities differed between pre and postmenopaused women with post-menopausal having more abnormal mammograms (p&lt;0.0005) although the incidence of breast cancer (BC) was similar. In women with BC, one underwent total mastectomy while three had surgical removal of the lesion with one woman requiring chemotherapy. Two other cases were treated with tamoxifen.</p>\r\n<p>\r\n	The overall detection rate for BC was less than 0.013. The rate of BC among premenopaused was 50% higher than in postmenopaused women (0.015 versus 0.010) although it was not statistically significant. The distribution of abnormal mammograms between the ethnic groups showed no significant difference. Likewise, the rate of BC between Malays (0.016) and Chinese (0.012) was not significant statistically. Among Malay women, the BC rate was twice as high among the postmenopaused compared to pre-menopaused while the reverse was observed among the Chinese.</p>\r\n<p>\r\n	Most of the breast biopsies reported benign lesions and subjects were advised to have regular follow ups. A repeat mammography was advised for those with microcalcifications (n=12), macrocalci-fication (n =1), benign nodules (n =2) and other structural abnormalities on the first mammogram. More premenopausal women (n=10) had dense or fibrous breasts compared to the postmenopaused although the difference was not significant.</p>\r\n<p>\r\n	The occurrence of abnormal mammograms was positively associated with education (rho=0.120, p=0.009) and negatively with pregnancy (rho=-0.108, p=0.019). Pregnancy was categorized as never pregnant or pregnant, being defined as one or more pregnancies with or without live birth. Logistic regression analysis of both factors demonstrated a R<sup>2</sup>=0.037 (p=0.013) but only education was signifi-cant in the final model (R=0.115, p=0.018).</p>\r\n<div>\r\n	<p>\r\n		Findings from this study seemed to concur with the Malaysian epidemiological data that the incidence rate of BC peaked earlier with a higher incidence among premenopausal women. Screening for breast abnormalities in perimenopausal women at and above 45 years old was definitely indicated from these findings in order to provide women with quality health.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		The subjects in this study with an urban ethnic distribution, higher income and better education did not reflect middle aged Malaysian women in general. Furthermore, women who were highly motivated and health conscious volunteered to participate in this study. They could likely possess a better health status with lower risk factors for development of diseases, a bias of the recruitment process. Nevertheless, the data from this large study could be a useful reference for future research into the breast and cervical cancer incidences in this often neglected age group.</p>\r\n	<p>\r\n		<strong><em>Cervical cancer (CC)</em></strong></p>\r\n	<p>\r\n		Postmenopausal women presented with a 2.8 fold higher CIN grade 1 or 2 compared to the premenopaused, in concordance with epidemiological evidence. The detection rate of CIN was 1.3%, similar to the pickup rate of 1.4-1.8% in women age 40 years and above, seen at UHKL over a 5 year period (Lim 2002). Generally, the peak incidence of invasive cancer of the cervix was in the fifth and sixth decades of life (Parkin et al 1999; Wright et al 2002).</p>\r\n	<p>\r\n		Unsatisfactory smears were 2.8 fold more among the postmenopaused. This was attributed to atrophic changes of the vagina and cervix resulting in inadequate cells for cytology reporting. Improvement of the smear could be made by wetting the cervix with normal saline prior to the procedure. Although premenopausal women had 2.5 fold higher infection rate compared to postmenopaused, a similar HPV infection rate was observed in both. HPV had been unequivocally recognized as the oncogenic agent causing dysplasia and CC in one third of all infected women (Parkin et al 1999; Reeves at al 1989; Waxman 2005).</p>\r\n	<p>\r\n		Pathology data from HUKM supported the overall increasing trend of CC as CC mortality rates in Malaysia declined from 1985 to 1993 and then increased in 1997 (NCR 2002). The data also indicate that CC was manifesting at an earlier age. This could be due to lifestyle changes or earlier sexual activity in the younger group. Increasing incidence rate could possibly reflect higher public awareness of the disease and the initiative to seek medical attention. But a more likely scenario is the increasing prevalence of HPV infection, an aetiological factor in CC and the futility of sampling preinvasive lesions for adenocarcinoma (Adeeb et al 2007a).</p>\r\n	<p>\r\n		Overall the data supported screening to be provided to all women particularly aged 40 and above as CC increased dramatically in women as they approached middle age. The study findings supported vigorous screening for CC which should be implemented in postmenopausal women as the incidence of malignancy was higher. Studies showed that approximately half of all women who died of cervical cancer had not had a Pap smear performed in the past five years. In countries where cervical smear screening was introduced, mortality rates from cervical cancer were reduced by 80% (Waxman 2005).</p>\r\n	<p>\r\n		<strong><em>Breast Cancer (BC)</em></strong></p>\r\n	<p>\r\n		In this study, the rate of new BC cases was 6 per 471 women or 1273.9 per hundred thousand. For comparison, the incidence rate was 299.5 and 123.3 per hundred thousand for USA and Hong Kong respectively. It is recognised that case detection rate at the initiation of a screening program (i.e. first screening) tended to inflate the figures and did not reflect the true incidence rate of the general population (Leung et al 2002). From randomised trials, the highest detection rate at screening was 350 per hundred thousand in women aged 50 years and above (Miller et al 2000). Locally, the pick-up rate for new cases of breast diseases seen was 3.1% (Yip et al 1995). The possible explanation for the high figure in this study was the recruitment process whereby the subjects were self selected. These women were more health conscious and took the opportunity to avail themselves for a health examination upon participation.</p>\r\n</div>\r\n<p>\r\n	In the developed world, BC was seen mainly in the postmenopausal group (Bush et al 2001; Chlebowski et al 2003). In Malaysia, the peak prevalence age group was between 40-49 years old and the median age was 50 years, a phenomenon also observed in this study. Similarly, benign breast diseases formed the majority of breast diseases seen (Yip et al 1995). Data from the population-based Singapore Cancer Registry for 1968-1992 revealed an average annual increase of 3.6% over the 25-year period for all women, from 20.2 per 100,000 women in 1968-1972 to 38.8 per 100,000 in 1988-1992. The rate of increase was highest in Malays (4.4%) and lowest in Indians (1.4%).</p>\r\n<p>\r\n	From the small number of cases in this study, the significant predictor of abnormal mammograms was education. The association of education to abnormal mammograms emphasised the relationship that educated women will actively seek healthcare (Cheah &amp; Looi 1999). Population trends indicated that incidence rates were likely to increase sharply in the future and suggested the role of demographic and lifestyle changes as possible risk factors (Seow et al 1996). Epidemiological factors associated with BC included increasing BMI, low physical activity, reproductive factors, diet, alcohol intake and hormone replacement therapy (Brewster &amp; Helzlsouer 2001). Lifestyle practices such as breastfeeding offered a protection against breast cancer particularly among Chinese women in Klang valley, odds ratio 4.43 after adjustment for confounders (Hejar et al 2004).</p>\r\n<p>\r\n	Asian culture and women&rsquo;s mentality constituted a major obstacle in promoting prevention of BC. Women were not empowered with the knowledge they need to seek preventive screening, and older women were difficult to reach or given information because they were unlikely to visit maternal-child health or family planning clinics (Anon 1997).&nbsp; Studies had observed more advanced and larger tumours among Malays than other ethnic groups, mainly due to a later stage at presentation. The delay in presentation was attributed to a strong belief in traditional medicine, the negative perception of the disease, poverty and poor education, coupled with fear and denial (Hisham &amp; Yip 2004). Cultural taboos prevented Malay women from examining their own bodies which resulted in denial of preventive care. Furthermore women fear their husbands would leave them after a mastectomy (Anon 1997). The availability of good health care and socioeconomic status of rural communities with limited health care facilities, lower education and poor income also influenced the presentation and detection of cancers. A higher risk for developing CC and prevalence pattern in ethnic Indians were partly a result of their socioeconomic deprivation (Cheah &amp; Looi 1999).</p>\r\n<p>\r\n	It should be made known that women with a first degree family history of BC had a 1.7 fold increased risk of ovarian cancer (OC); an association with reciprocal ramifications. An increased risk of 2.2 fold was seen in women with a first degree relative affected with OC. Corresponding risks of 7.1 to 10.7 fold were observed for those with two or three affected relatives. Screening a population for OC with at least one first degree affected relative gave a prevalence of 3.8/1000 women. Screening women with BC/OC diseases gave a prevalence of 50/1000 (Collins et al 1998).</p>\r\n<p>\r\n	Malaysian women above 40 years should be offered and encouraged to have mammogram screening as an earlier peak prevalent age for BC was observed. Additionally, postmenopaused women were noted to have abnormal mammogram of a graver nature with 2.5 fold more requiring biopsies and 30% more needed regular follow ups. For this reason, a bi-pronged approach could be advocated whereby younger women aged 40 years and above should have their first mammogram followed by a 5 year interval when the outcome was normal. Emphasis should be given for more frequent mammograms in those above 50 years old, possibly annually.</p>\r\n<p>\r\n	In summary, cancers of the cervix and breast formed two fifths of all malignancies in women (NCR 2002) but more importantly, both diseases were amenable to measures for early detection and treatment. Hence in cancer prevention, health education to increase awareness, screening and early treatment could reduce the burden of both diseases (Lim 2002). The study supports screening for CC in postmenopausal women as the incidence of CIN and HPV was higher whereas mammogram screening should be offered and encouraged in women above 40 years as an earlier peak prevalent age for BC was observed. Pap smear, ultrasound of the pelvis and mammogram should be used synergistically with clinical judgment after completing a detailed history of risk factors and physical examination. With the wide use of pelvic ultrasound for imaging, endometrial thickness and ovarian volume normogram (Adeeb et al 2007b) should be made available for the local population to enable the clinician to tailor subsequent management and counselling.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors gratefully acknowledge and thank the following for their assistance &ndash; Prof Sharifah Noor Akmal, Cytopathology Unit, Dept of Pathology for cervical cancer pathology data. We wish to thank Prof Nafikudin Hj Mahmud, Siti Zakiah Othman, Radiology Unit, Dept of Radiology for assisting in the study. Funding for the project came from USM Top Down Program IRPA No:06-02-05-9002 for which we would like to thank the program directors, Dato&rsquo; Prof Mafauzy Mohamed and Dato&rsquo; Prof Mustafa Embong.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(413, 410, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Anaplastic large cell lymphoma (ALCL) is a rare tumour, accounting for approximately 3% of adult non-Hodgkin lymphomas (Delsol et al. 2001).Three entities of ALCL have been identified: primary systemic ALK+ ALCL, primary systemic ALK- ALCL, and primary cutaneous ALCL (Stein et al. 2000). Primary systemic ALCL frequently involves both lymph nodes and extranodal sites. Extranodal sites commonly involve the skin (21%), bone (17%), soft tissues (17%), lung (11%) and liver (8%). Other rare sites include gut, central nervous system, parotid gland, pancreas and kidney (Delsol et al. 2001; Stein et al. 2000; Yamamoto et al. 2003; Cohen et al. 2003 &amp; Venizelos et al. 2003).</p>\r\n<p>\r\n	The majority of soft tissue lymphomas are of non-Hodgkin B-cell type, including diffuse large-cell lymphoma, small lymphocytic cell lymphoma and follicular lymphoma (Knowles et al. 2003). Anaplastic large cell lymphoma is a T-cell or null cell phenotype, but shows evidence of T-cell genotype (Delsol et al. 2001).</p>\r\n<p>\r\n	We report a case of a 44-year-old female presenting with ALCL in the soft tissue.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 44 year-old lady presented with a left iliac fossa mass of 3 weeks duration which was rapidly increasing in size recently. For the past 3 years she had constipation but she was otherwise asymptomatic. She had no other medical history or previous surgery.</p>\r\n<p>\r\n	On initial examination, there was a superficial soft to firm swelling which was slightly tender, measuring 4 x 4 cm in the abdomen. Ultrasound showed a fairly well defined inhomogenous soft tissue mass, measuring 4 x 4 x 2.6cm in the deep subcutaneous region in the left iliac fossa (Figure 1). Some posterior echo enhance-ment was present.</p>\r\n<p>\r\n	The following week the overlying skin was reddish in colour, associated with mild pain which became much worse later. She was afebrile, conscious and alert. The mass was slightly larger, measuring 5 x 6 cm. It was firm, mobile, fluctuant, slightly tender and the overlying skin was inflamed. The impression at this time was an anterior abdominal abscess and the patient was given a course of antibiotics. Aspiration yielded blood stained serous fluid.</p>\r\n<p>\r\n	Two weeks later she was readmitted to another hospital for shortness of breath and cough. A computed tomography (CT) scan showed a solitary right broncho-pulmonary mass in the upper lobe suggestive of bronchogenic carcinoma.</p>\r\n<p>\r\n	There was no mediastinal lymph node or endobronchial tumor seen but there were bilateral axillary lymph nodes, measuring one cm in diameter (no biopsy was available).</p>\r\n<p>\r\n	Excision biopsy of the anterior abdominal mass was performed and diagnosed histologically as lymphoplasmacytic lym-phoma by the pathologist in the other hospital. The patient returned to Hospital universiti Kebangsaan Malaysia (HUKM) for further treatment. We received a block of the paraffin-embedded specimen and 4 &mu;m thick sections were made. Histologic examination showed a circumscribed tumour mass infiltrated by large pleo-morphic lymphoid cells displaying marked nuclear atypia, vesicular nuclei, and kidney-shaped and abundant cytoplasm (Fig. 2) in a background of macrophages, lymphocytes and plasma cells. Mitotic figures and areas of necrosis were present. Immunohistochemistry performed on formalin-fixed, paraffin-embedded sections using the avidin-biotin complex technique (Vector, Burlingame, CA) showed that the neoplastic cells expressed ALK protein in the nuclei and cytoplasm, and EMA. The neoplastic cells were negative for CD45RO, CD3, CD20 and CD79&alpha; (Table 1). The diagnosis was of ALK-positive anaplastic large cell lymphoma (common variant).</p>\r\n<p>\r\n	Fluorescence <em>in situ</em> hybridization (FISH) analysis was performed using the LSI ALK breakpoint-spanning and flanking dual-colour DNAprobes (Vysis, Inc., Downers Grove, IL) (Bridgeet al. 2001). A t(2;5) or other chromosome rearrangement at the 2p23 ALK breakpoint region would be indicated by one orange and one green signal, while the native ALK region will remain as an orange/green fusion signal (1O1G1F), [Fig.3].</p>\r\n<p>\r\n	She was at Stage IIA of the disease. The lactate dehydrogenese was raised to 535 U/l (Normal = 211-423 U/L) but there was no bone marrow infiltration. She was treated with CHOP (cyclophosphamide, vincristine, allopurinol and prednisolone) chemotherapy. One year later she developed pulmonary tuberculosis and was treated with anti-tuberculous drugs. She completed six cycles of CHOP chemo-therapy and remains healthy at follow-up.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Lymphoma presenting as a soft tissue mass is rare and may thus be confused with the more common soft tissue sarcoma (STS) (Knowles et al. 2003 &amp; Damron et al. 1999).</p>\r\n<p>\r\n	Damron et al (1999) reported that clinical and radiographic features that favor extranodal soft tissue lymphoma over sarcoma include pain and tenderness, lymphadenopathy (particularly when con-fluent radiologically), ipsilateral extremity swelling and elevated lactate dehydro-genese (Damron et al. 1999). Another recent article suggested computed tomo-graphy scanning and core biopsy for such cases where the latter established a diagnosis in 13 out of 17 patients (sensitivity 93%) (Knowles et al. 2003). Our patient had pain, and tenderness. Lactate dehydrogenese was elevated.</p>\r\n<p>\r\n	Clinically, ALK-positive lymphoma mostly occur in children and young adults with a male predominance. It usually present as an aggressive, stage III-IV disease, frequently associated with systemic symptoms and extranodal involvement (Falini et al. 1999). In contrast, ALK-negative cases occur in older individuals (mean age, 44.33 years) and show lower male/female ratio (0.9) as well as a lower incidence of stage III-IV disease and extranodal involvement at presentation. Our patient was in the older age group and was ALK-positive.</p>\r\n<p>\r\n	The commonest sites for STS and extra-nodal lymphomas are the lower limbs, especially in the thigh (25%), retro-peritoneum (16%), and upper limb (11%) (Knowles et al. 2003). Other unusual sites include the erector spinae muscle, the supraclavicular fossa and surrounding the wing of the ileum. Our patient presented in the left iliac fossa.</p>\r\n<p>\r\n	There was no confirmation of nodal involvement by histology in this patient, but the expression of ALK in an extranodal site favours systemic ALCL with (primary) nodal involvement (ten Berge et al. 2000). The definition of primary extra-nodal soft-tissue lymphoma is the presence of soft tissue lymphoma in a region not typically considered to be rich in lymph nodes (Knowles et al. 2003).</p>\r\n<p>\r\n	ALCL contain cells with eccentric, horse shoe- or kidney-shaped nuclei often with an eosinophilic region near the nucleus, referred to as hallmark cells (Delsol et al. 2001).The application of these stains, CD30 and ALK immunostaining is helpful in the diagnosis of ALCL. ALK expression can also be detected in inflammatory myo-fibroblastic tumours (IMT) and a variety of other non-inflammatory myofibroblastic tumours and soft tissue tumours (Li et al. 2004). Most cases, except the inflam-matory myofibroblastic tumours, display low-level expression of ALK.</p>\r\n<p>\r\n	The most important prognostic indicator is ALK positivity, which has been associated with a favourable prognosis (Delsol et al. 2001). This patient is well till today.</p>\r\n<p>\r\n	The most frequent alteration of ALCL is the translocation, t(2;5)(p23;q35)), between the ALK gene on chromosome 2 and the nucleophosmin (<em>NPM</em>) gene on chro-mosome 5 (Delsol et al. 2001). The classic t(2:5) leads to positive staining for ALK in both the nucleolus and the cytoplasm. Similar findings have been reported in other studies (Delsol et al. 2001).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	ALCL presenting as a soft tissue tumour is rare and it is important that it be included in the differential diagnosis.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors thanked Professor H.K. Muller Hermelink of the Institute of Pathology, University of Wurzburg, Germany, for confirmation of the diagnosis during the Precongress tutorial; a comprehensive course &lsquo;lymph node diagnosis&rsquo; at the Third IAP Asia Pacific Meeting, Bangkok, Thailand, January, 2003, and also Encik Rosli Nasir of Hospital Universiti Kebangsaan Malaysia for the photographs.</p>\r\n', 1),
(414, 411, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The demand for the healthcare system to deliver high quality services whilst con&shy;taining expenditure, has forced health ser&shy;vice providers to examine and re&shy;structure the clinical management sys&shy;tems. There is a need for healthcare or&shy;ganizations to per&shy;sistently examine pa&shy;tient care processes, deliver superior quality care, achieve posi&shy;tive clinical and better financial outcomes (Coffey et al. 1992, Pearson et al. 1995, Jones et al. 1999).</p>\r\n<p>\r\n	The focus of health services has shifted towards controlled clinical management strategies to make healthcare organiza&shy;tions more efficient without compromising the quality of care delivered. There are increasing demands for more appropri&shy;ate use of technologies, more coordi&shy;nated care and more enhanced care given to the pa&shy;tient. Qualities of care and cost contain&shy;ment are debatable and are the foremost and principal agenda on the policy in many countries today (Cheah 2000).</p>\r\n<p>\r\n	The Ministry of Health, Malaysia has im&shy;plemented several quality assurance pro&shy;grams in government hospitals such as peri-operative morbidity review (POMR), clinical audit, risk management and inci&shy;dent reporting (Ministry of Health Malay&shy;sia 1998). All these programs are very impor&shy;tant to maintain the quality of healthcare in government hospitals.</p>\r\n<p>\r\n	Clinical pathway has been introduced in many hospitals and has been accepted as a beneficial tool. The CP assists the healthcare organization in providing bet&shy;ter quality and coordinating services within scarce resources (Dowsey et al. 1999, Hoffart &amp; Kuckelman 2000, Kohn et al. 2000, Every et al. 2000). Clinical pathway differs from clinical guidelines, protocol and algorithms. Clinical guide&shy;lines are consen&shy;sus statements that are systematically de&shy;veloped to assist practi&shy;tioners in making patient management decisions related to specific clinical cir&shy;cumstances (Field &amp; Lohr 1990, Dwyer 1998, Gaddis et al. 2007). Protocols are treatments that were recommended based on guidelines.</p>\r\n<p>\r\n	What is clinical pathway? Clinical path&shy;way is a methodology for mutual decision making, and is an organization of care for a well-defined group of patients during a well-defined period. (European Pathway Association, Slovenia Board Meeting, Dec 2005). The pathway can also be de&shy;fined as a multidisciplinary plan of care based on best clinical practice for specific groups of patients with a particular diag&shy;nosis designed to minimize delays, opti&shy;mise resource utilization and to maximize the quality of care (Pearson et al. 1995, Campbell et al. 1998, Panella et al. 2003). The aim of a care pathway is to enhance the quality of care and cost control by im&shy;proving patient outcomes, promoting pa&shy;tient safety, increasing pa&shy;tient satisfaction, and optimising the use of resources(European Pathway Association, Slovenia Board Meeting, Dec 2005).</p>\r\n<p>\r\n	Clinical pathway is also known as criti&shy;cal pathway, care map or integrated care pathway. It forms all or part of the clinical record, document the care given and faci&shy;litates the evaluation of outcomes for conti&shy;nuous quality improvement. The CP was developed by multi-professional teams, consist of physicians (family prac&shy;titioners to specialist), nurses, physiothe&shy;rapist, phar&shy;macist, social workers and managers (Pearson et al. 1995, Gregor et al. 1996, Mabrey et al. 1997, Campbell et al. 1998, Healy et al. 1998, Hill 1998, Pritts et al. 1999, Cheah 2000, Huerta et al. 2001, Pearson et al. 2001, Uchiyama et al. 2002, Panella et al. 2003, Lee &amp; Anderson 2006). The pathway was able to facilitate the use of clinical practice guidelines by the multi&shy;disciplinary team, as pathway was locally agreed and avail&shy;able in the patients&rsquo; record (Kitcher &amp; Bundred 1998).&nbsp;</p>\r\n<p>\r\n	The CP was first developed in the 1950s to coordinate multiple contractors or per&shy;sons in a project by identifying the key se&shy;quence of events or critical point which would drive the timeline of the overall project (Pearson et al. 1995). In healthcare system the CP was first de&shy;veloped and applied in the 1980s at the New England Medical center by Karen Zander and Kathlen Bower (Pearson et al. 1995). The development of CP was in response to the initial Diagnostic Related Group (DRG) based reimbursement system (Pearson et al. 1995, Luttman 2000). The CPs were introduced in the early 1990s in the United Kingdom and the United State of America (Kitcher &amp; Bundred 1998, Lutman 2000)</p>\r\n<p>\r\n	The implementation of CP is highlighted in the current health care sector because of its similarity to the descriptions of case-mix system which addresses the same clinical characteristics and re&shy;sources involved (Cheah 2000). The de&shy;velopment and im&shy;plementation of CP can be applied in ho&shy;mogenous population, high volume cases, high risk cases and common cases. The CP can also be ap&shy;plied in clinical protocols and guidelines, by committed and account&shy;able health&shy;care providers, health managers and government. The aim of this article was to highlight the benefits of CP in im&shy;prov&shy;ing healthcare quality and cost control.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	For selection of relevant articles, the search was focused on the terms &lsquo;clinical pathway&rsquo;, &lsquo;care path&rsquo; and integrated care pathway. Most studies were conducted in the United States, Canada, Australia and United Kingdom. Only few studies were conducted in Asia.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>What can be measured from clinical pathway?</em></p>\r\n<p>\r\n	Continuous evaluation and follow up are essential to establish a good CP. The ef&shy;fect of clinical pathway can be measured in five domains; clinical outcome, service, team, process and finance (Herck et al. 2004).The indicators for clinical outcome domain are number of readmissions, com&shy;plications, mortality, number of re&shy;lapses without admission, time until extu&shy;bation, time until normal food intake, quality of life, number of infections, level of pain and length of stay (Kucenic &amp; Meyers 2000, Johnson et al. 2000, Huerta 2001, Benson et al. 2001, Darer et al. 2002, Hoffart &amp; Kuckelman 2002, Kinsman 2004).</p>\r\n<p>\r\n	For process domain, the indicators are analysis of variance, number of clinical ex&shy;aminations (laboratories, radiology), com&shy;pleteness and quality of documenta&shy;tion, time schedules , planning and ana&shy;lysis of process flow (Johnson et al. 2000, Kucenic &amp; Meyers 2000, Huerta 2001, Benson et al. 2001, Hoffart &amp; Kuckelman 2002, Darer et al. 2002, Kinsman 2004).</p>\r\n<p>\r\n	The indicators for team domain are team communication, team satisfaction, know&shy;ledge and competence, recognition and appreciation of roles, autonomy of physi&shy;cian, self confidence and influence on un&shy;derstanding (Johnson et al. 2000, Hoffart &amp; Kuckelman 2002, Kinsman 2004).</p>\r\n<p>\r\n	For financial domain the indicators are length of stay, medical, consumption of resources and number of staff .( Kucenic &amp; Meyers 2000, Kelly 2000, Johnson et al. 2000, Benson et al. 2001, Huerta 2001, Hoffart &amp; Kuckelman 2002, Darer et al. 2002, Kinsman 2004). Finally, the service indicators are patient and family satisfac&shy;tion (Hoffart &amp; Kuckelman 2002, Darer et al. 2002).</p>\r\n<p>\r\n	Herck et al. (2004) evaluated the effect of the implementation of the clinical pathways and found that the majority of the articles reviewed between years 2000 to 2002 had positive effects on three do&shy;mains namely process, team and finan&shy;cial domain (Table 1).</p>\r\n<p>\r\n	<em>Benefits of clinical pathway</em></p>\r\n<p>\r\n	The majority of literature reviewed con&shy;cluded that there were positive effects in implementing CP. According to more than 80% of the authors, the CP is found to be significant in reducing length of stay and medical cost. The articles include man&shy;agement in patient asthma in pae&shy;diatric cases (Banasiak &amp; Oliver 2004, Cheney et al. 2005), management on uncomplicated acute myocardial infarc&shy;tion (Nichol 1997, Cheah 2000, Kucenic &amp; Meyers 2000, Bahit et al. 2002, Zevola et al. 2002, Lee &amp; Anderson 2006), man&shy;agement of conges&shy;tive cardiac failure (Hogkin 2001, Ranjan et al. 2003), man&shy;agement of pneumonia (Estrada et al. 2000, Loeb et al. 2006) and management of nephrology cases (Benson et al. 2001).The CP is also useful in man&shy;aging surgical cases such as knee joint man&shy;agement (Gregor et al. 1996, Macario et al. 1998, Dowsey et al. 1999, Pearson et al. 2001, Panella et al. 2003, Pennington et al. 2003, Brunenberg et al. 2005, Walter 2005, Xu et al. (2008),manage&shy;ment frac&shy;ture neck of femur (Choong et al. 2000), management of bowel resec&shy;tion (Pritts et al. 1997), laparoscopic sur&shy;gery (Huerta et al. 2001, Uchiyama 2002) fluid manage&shy;ment in femoral neck frac&shy;ture (Davidson 2007) and patients who had undergone total laryngectomy (Hanna et al. 1999).</p>\r\n<p>\r\n	Standardized processes and better coor&shy;dination in CP have improved patient care among clinicians and managers (Pearson et al. 1995, Gregor et al. 1996, Mabrey et al. 1997, Healy et al. 1998, Pritts et al. 1999, Cheah 2000, Huerta et al. 2001, Pearson et al. 2001, Uchiyama et al. 2002, Panella et al. 2003, Lee &amp; Anderson 2006). The CP also improved clinical outcomes through the variance management system (Luttman 2000, Santoso et al. 2002).</p>\r\n<p>\r\n	The CP was able to maximize the effi&shy;cient use of resources in reducing unne&shy;cessary documentation (Pearson et al. 1995, Kucenic &amp; Meyers 2000, Kelly 2000, Johnson et al. 2000, Benson et al. 2001, Huerta 2001, Hoffart &amp; Kuckelman 2002, Darer et al. 2002, Kinsman 2004). Most of the articles concluded that one of the measures to control costs and to as&shy;sure quality is the use of a case-ma&shy;naged CP model. The authors also re&shy;vealed CP is useful for costing services (pricing and contracting) (Field &amp; Lohr 1990, Gregor et al. 1996, Healy 1998, Luttman 2000, Huerta et al. 2001)</p>\r\n<p>\r\n	According to Zander et al. (1988), the major reasons for developing CP are to improve patient care by improving the quality of patient care through consistent management, identifying and measuring improvements in patient care, and mea&shy;suring outcome. The CP has also im&shy;proved communication and collaboration among all disciplines, enhanced the dis&shy;charge coor&shy;dination process and estab&shy;lished protocols to all members of the health care team. CP also can be used to support clinical audit and risk manage&shy;ment (Zander et al. 1988, Dowsey et al. 1999, Cheah 2000, Benson et al. 2001, Darer et al. 2002, Panella et al. 2003, Xu et al. 2008)</p>\r\n<p>\r\n	In the articles reviewed, only a few stu&shy;dies were conducted in an Asia health care setting. In Singapore, a study con&shy;ducted by Cheah (2000) at a General Hospital dis&shy;covered the average length of stay for un&shy;complicated acute myocardial infarction using CP was significantly re&shy;duced. There was no significant increase in hospital mortality, complication rate and no read&shy;mission rate at six months after discharge. He concluded that CP was able to improve care processes through better collaboration among healthcare professionals and im&shy;proved work systems (Cheah 2000). Santoso et al. (2002) had developed a mastectomy clinical pathway for breast cancer pa&shy;tients at the National University Hospital of Singapore and the results showed that the implementation of the CP had im&shy;proved patients&rsquo; treatment and out&shy;come. The medical cost and length of hos&shy;pital stay also was reduced. Similar finding by Xu et al. (2008) shown that the use of knee pathway had led to a significant de&shy;crease in the length of stay, complication rates and early mobilization among 1663 patients who underwent total knee re&shy;placement (tkr) in a tertiary institution in Singapore.</p>\r\n<p>\r\n	In Taiwan, a study carried out by Liao et al. (1998) at Tsu Chi General Hospital found that CP was able to decrease re&shy;source consumption, control medical ex&shy;penditure and decrease the number of pro&shy;cedures performed. They also demon&shy;strated no changes in clinical outcomes and complication rates in patients who un&shy;derwent transurethral resection of the prostate. In another study conducted by Chang &amp; Lin (2003) they also found the implementation of the CP for patients who underwent vaginal hysterectomy had im&shy;proved health care outcomes, de&shy;creased length of hospital stay and ad&shy;mission fees. The authors concluded that CP is a good policy for cost management and in en&shy;hancing the quality of care.</p>\r\n<p>\r\n	Paiboon (2006) in his study conducted at Taksin Hospital in Thailand also found the implementation of clinical pathway among type 2 diabetic patients had re&shy;duced the length of hospital stay and de&shy;creased readmission rate of recurrent hypoglycae&shy;mia.</p>\r\n<p>\r\n	Many studies on CP have been carried out in United States (US) and United Kingdom (UK) (Wigfield &amp; Boon 1996, Chang et al. 2006). The implementation of CP was reported to be high in US and UK with the apparent outcomes such as re&shy;duction in the incidence of complica&shy;tions, medical cost and improved patient satis&shy;faction (Chang et al. 2006 and Wigfield &amp; Boon 1996).</p>\r\n<p>\r\n	Ranjan et al. (2003) had carried out a study among congestive heart failure (CHF) patients at hospitals in United States and the result showed that the patients as&shy;signed to the CHF CP had a shorter length of stay and reduced hos&shy;pital charges com&shy;pared with those who were on the usual care. The quality of care delivered (as measured by adminis&shy;tration of ACE inhibi&shy;tors) was not com&shy;promised by the reduc&shy;tion in length of hospital stay in patients on the clinical pathway. Further there was a significant saving of US2,500 per patient and US750,000 per year in CHF treatment (Ranjan et al. 2003)&nbsp;</p>\r\n<p>\r\n	A study conducted by Pearson et al. (2001) also found with the implementa&shy;tion of the CP, the length of stay de&shy;creased 21% for total knee replacement, 9 % for CABG surgery, 7% for thoracic surgery, 5% for hysterectomy and 3% for colect&shy;omy. In 1999, Pritts et al. con&shy;ducted a study at University of Cincinnati Medical Center, Ohio and showed a sig&shy;nificant de&shy;crease in length of stay and medical cost in the pathway groups who underwent bowel resection.</p>\r\n<p>\r\n	Several studies also found improve&shy;ment of interdisciplinary cooperation, staff satis&shy;faction (Mabrey et al. 1997, Maxey 1997, Hanna et al. 1999, Jacavone et al. 1999)and also patient satisfaction (Jacavone et al. 1999, Walter 2005).</p>\r\n<p>\r\n	About 40.0% of the authors discovered the clinical outcomes such as readmis&shy;sion, mortality and complication rates showed no significant difference between patients on clinical pathway and patients on usual care (Nichol et al. 1997, Macario et al. 1998, Jones et al. 1999, Choong et al. 2000, Farquhar 2000, Johnson et al. 2000, Kucenic &amp; Meyer 2000, Hogkin et al. 2001, Huerta et al. 2001, Uchiyama et al. 2002, Ranjan et al. 2003, Banasik &amp; Oliver 2004, Brunenberg et al. 2005, Lee &amp; Anderson 2006). Nevertheless, one third of the au&shy;thors found the readmission and compli&shy;ca&shy;tion rates were reduced in the clinical pathway group and there was significant difference compared to the non clinical pathway group (Dowsey et al. 1999, Estrada et al. 2000, Cheah 2000, Benson et al. 2001, Bahit et al. 2002, Darer et al. 2002, Ransom et al. 2002, Panella et al. 2003, Bestul et al. 2004, Cheney et al. 2005, Walter et al. 2005, Loeb et al. 2006, Paiboon 2006, Xu et al. 2008).</p>\r\n<p>\r\n	Even though CP has been approved as beneficial tools in improving quality and controlling healthcare cost, there are a few aspects and factors which require attention as revealed by nearly one third of the ar&shy;ticles reviewed (Gregor et al. 1996, Pritts et al. 1997, Macario et al. 1998, Dowsey et al. 1999, Choong et al. 2000, Cheah 2000, Pearson et al. 2001, Hogkin 2001, Uchiyama 2002, Panella et al. 2003, Ranjan et al. 2003, Brunenberg et al. 2005, Walter 2005, Lee &amp; Anderson 2006). The aspects that require attention are the un&shy;derstanding, implementation, accountabil&shy;ity, the system and the ad&shy;ministrative sup&shy;port of the CP.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	As a conclusion, the majority of literature reviewed concluded that there were posi&shy;tive effects in implementing CP. The CP was not only found to be significant in re&shy;ducing length of stay and medical cost but is also found to support the introduc&shy;tion of evidence based medicine, clinical audit, multi disciplinary communication, teamwork and care planning. In addition, the CP is able to support the continuity and coordi&shy;nation of care across different clinical dis&shy;ciplines, reduce variances in patient care (by promoting standardiza&shy;tion), help im&shy;prove patient documentation and optimize the management of re&shy;sources. Even though CP had been shown to improve quality and control of healthcare costs, its successful imple&shy;mentation would require full commitment andparticipation from all health care pro&shy;viders and healthcare man&shy;agers.</p>\r\n', 1),
(415, 412, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Osteoporosis is a condition of decreased bone mass whichleads to fragile bones with increased risk of fractures. It has affected 75 million people in Europe, USA and Japan (EFFO &amp; NOF 1997). One in 3 women and 1 in 5 men over 50 years old will experience osteoporotic fractures (Melton et al. 1998, Kanis et al. 2000, Randell et al. 1995, Gullberg et al. 1997). It is projected that more than 50% of all osteoporotic hip fractures will occur in Asia by the year 2050 (Cooper et al. 1992). Post menopausal osteoporosis, which occurs in estrogen-deficient wo-men, is the commonest form of osteo&shy;porosis. There is increasing evidence that oxidative stress may be partly res&shy;ponsible for post-menopausal osteoporo&shy;sis. Estrogen itself is an antioxidant with radical-scavenging properties and its de&shy;ficiency has been associated with oxida&shy;tive stress (Dubey et al. 2000). Oxidative stress has been shown to stimulate os&shy;teoclast differentiation and function (Garrett et al. 1990) and inhibit osteoblast differentiation (Bai et al. 2004). Ferric nitrilotiracetate, an oxidizing agent which causes oxidative stress was shown to reduce bone calcium content in young growing rats (Yee &amp; Ima Nirwana 1998), increase osteoclast numbers and reduce bone volume and thickness (Ahmad et al. 2005).</p>\r\n<p>\r\n	Many human studies have linked oxi&shy;dative stress to osteoporosis. In subjects with osteoporosis, the malondialdehyde (MDA) levels were increased while the antioxidant enzymes, superoxide dismu&shy;tase and glutathion peroxidase were re&shy;duced compared to the control group (Sontakke et al. 2002). While in another recent study, osteoporotic women were found to have high MDA levels and low catalase and gluthatione peroxidase ac&shy;tivities (Ozgocmen et al. 2007). Elderly women with osteoporosis were found to have lower endogenous and exogenous anti-oxidants (Maggio et al. 2003). A population based study found an associ&shy;ation between the increases of prostag&shy;landin F<sub>2</sub><sub>a</sub>, a bio-marker of oxidative stress to reduction in bone mineral den-sity (Basu et al. 2001).</p>\r\n<p>\r\n	With all the evidence pointing to the in&shy;volvement of oxidative stress in osteopo&shy;rosis, studies on the ability of potent anti-oxidants like vitamin E to protect bone from osteoporosis in estrogen deficient women need to be carried out. It is also important to determine if the benefit of vitamin E intake would extend to better bone strength in order to reduce the risk of osteoporotic fracture. A biomechanical test is a useful tool to evaluate bone strength which is associated with sus&shy;ceptibility to fracture. It can only be car&shy;ried out in animal models as bone sam&shy;ples need to be subjected to force until they break to get the accurate result. Previous studies have used the rat as a standard rodent model for bone biome&shy;chanical studies (Ferretti et al. 1993, Kimmel 1996, Peng et al. 1994, Raab et al. 1990, Tuukkanen et al. 1994).</p>\r\n<p>\r\n	Vitamin E is a fat soluble vitamin with a chain&ndash;breaking ability. There are two forms of vitamin E, tocopherol and toco&shy;trienol which can be further subdivided into a, b, gand d. Tocotrienol is a unique vitamin E which is abundant in palm oil of <em>Elaeis guineensis</em> species. Tocotrienol has better antioxidant ability compared to tocopherol because of its higher recycling efficacy (Serbinova et al. 1991, Kamat et al. 1997). Vitamin E is thought to protect bone via its antioxidant properties by helping the internal anti-oxidative de&shy;fense against free-radicals and sca&shy;venging the lipid peroxidation radicals, therefore preventing oxidative stress.</p>\r\n<p>\r\n	The aim of the study is to determine the protective effects of palm tocotrienol on the oxidative status and bone bio-me&shy;chanical strength of rat model at eight weeks post-ovariectomy.</p>\r\n<p>\r\n	<strong>METHODS</strong></p>\r\n<p>\r\n	Sixty-four female Wistar rats aged three months, weighing between 200-250 grams were housed in groups of three at 27&deg;C with 12:12 hour light-dark cycle. The rats were fed &lsquo;rat chow&rsquo; Gold Coin (Port Klang, Malaysia) and drinking water made available <em>ad libitum</em>.</p>\r\n<p>\r\n	There were eight rats in each group and they received the following treat&shy;ments: Group 1(Sham) rats were sham-operated. Group 2 (OVXC) rats were ovariectomised and given vehicle (olive oil) orally. Group 3 (OVX+ATF) rats were ovariectomised and given 60 mg/kg rat weight of a-tocopherol orally. Group 4 (OVX+PTT) rats were ovariectomised and given 60 mg/kg rat weight of palm tocotrienols orally.</p>\r\n<p>\r\n	Alpha-tocopherol acetate (Sigma, St. Louis, MO, USA) or palm tocotrienol mixture (Palm Oil Research Institute of Malaysia (PORIM) was diluted in olive oil ( Bertolli, Lucca, Italy) to obtain a con&shy;centration of 60 mg/kg rat weight, each in 0.1 ml volume. The diluted vitamin E was given orally to rats using gavage needle 6 days / week for three weeks. The palm tocotrienols has the following composi&shy;tion: a-tocotrienol (30.7%), g-tocotrienol (55.2%) and d-tocotrienol (14.1%). Olive oil was chosen as the diluents because it contains only 0.0051 % a-tocopherol and no tocotrienols.</p>\r\n<p>\r\n	Rats were bilaterally ovariectomised using dorsal approach under anaesthe&shy;sia. For sham-operated rats, the ovaries were identified but left intact. The rats were allowed to recover for two weeks before treatment was started.</p>\r\n<p>\r\n	Blood samples were collected into an EDTA tube from the retro-orbital vein of the anesthetized rats. The blood was centrifuged at 3000 rpm at 4&deg;C for 10 minutes and the plasma was stored at -70&deg;C. The erythrocytes were washed with normal saline, centrifuged at 3000 rpm for 10 minutes and stored at -20&deg;C. The left femurs were cleaned, wrapped in gauze soaked with phosphate-buffered solution and aluminum foil and stored at -70&deg;C.</p>\r\n<p>\r\n	The plasma malondialdehyde (MDA) levels were measured according to the method of Ledwozyw <em>et al. </em>(Ledwozyew et al. 1986). The MDA reacts with thi&shy;obarbituric acid (TBARS) to form a pin&shy;kish complex which was measured using a spectrophotometer (Shimadzu UV-160A, Kyoto, Japan) at a wavelength of 515 nm. The total protein in the plasma is measured according to the method of Lowry <em>et al.</em> (1951). The MDA value (nmol) is divided with the total protein in plasma (mg). The plasma glutathione peroxidase (GPX) and erythrocyte supe&shy;roxide dismutase (SOD) activities were measured by using RANSEL and RANSOD kits (RANDOX Lab Ltd, Co Antrim, UK) respectively.</p>\r\n<p>\r\n	The femurs were thawed at room tem&shy;perature one hour before the start of the biomechanical test. The femurs were subjected to three point bending configu&shy;ration using an Instron<sup>&reg; </sup>machine 5560 and were analysed using Bluehill<sup>&reg; </sup>2 soft&shy;ware (High Wycombe, UK). The data obtained were used to plot load versus displacement and stress versus strain graphs. Bone stiffness and Young&rsquo;s Modulus were then derived from the graphs.</p>\r\n<p>\r\n	The results were expressed as mean values &plusmn;S.E.M. Data analysis was per&shy;formed using Statistical Package for So&shy;cial Sciences (SPSS 12.0.1, Chicago, IL, USA) software. Statistical test used was ANOVA followed by Tukey&rsquo;s hsd for nor&shy;mally distributed data and Kruskal-Wallis and Mann-Whitney test for data that was not normally distributed. The results were presented as mean values &plusmn; standard error of the mean (SEM). This study was approved by the Universiti Kebangsaan Malaysia Animal Ethics Committee (UKMAEC).&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	There was a significantly higher level of MDA in the OVXC group compared to the Sham and OVX+PTT groups (Figure 1). This indicated that ovariectomy has led to an increase in lipid peroxidation activity. However, oral supplementation with palm tocotrienols at 60mg/kg was able to re&shy;duce the lipid peroxidation activity in the ovariectomised rats.&nbsp;</p>\r\n<p>\r\n	&nbsp;After treatment, the SOD levels of the OVXC group remained unchanged from the Sham group but were significantly lower than the OVX+ATF and OVX+PTT groups. Ovariectomy did not affect SOD levels but oral supplementation of 60mg/kg of &alpha;-tocopherol or palm toco&shy;trienols raised the SOD. The OVX+PTT group had higher levels of SOD com&shy;pared to the OVX+ATF group (Figure 2).</p>\r\n<p>\r\n	After treatment, the GPX levels of the OVXC group were significantly lower than the Sham and OVX+PTT groups. These results showed that ovariectomy had reduced the GPX level but oral sup&shy;plementation with palm tocotrienols pre&shy;vented the reduction in the GPX level (Figure 3).</p>\r\n<p>\r\n	As for the biomechanical parameters, a pattern was seen although not significant, whereby ovariectomy had weakened the femur ability to withstand load and stress, but all these were improved with vitamin E supplementations. Vitamin E supple&shy;mentations had also improved femur stiffness and Young&rsquo;s modulus value (Table 1).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The current treatments for post-meno&shy;pausal osteoporosis are estrogen re&shy;placement therapy, raloxifene, bisphos&shy;phonates and calcitonin. Alternative treatments using herbs and natural prod&shy;ucts have been suggested such as <em>Rehmania glutinosa</em>, royal jelly, <em>sumbucus williamsii </em>HANCE, chondroitin sulfate and fructo-oligosaccharides (Oh et al. 2003, Ha 2004, Devareddy et al. 2006, Xie et al 2005, Hidoka et al. 2006). Several studies have also shown the potential of certain vitamins as alternative treatments for osteoporosis. High doses of vitamin K<sub>2</sub> could partially prevent the bone loss in orchidectomised young rats by normalizing the raised bone resorption (Iwamato et al. 2003). An analogue of vitamin D<sub>3</sub> has been shown to promote bone growth in ovariectomised rats and cultured osteoblast (Shevde et al. 2002). Vitamin E especially palm tocotrienols were able to protect rat bones against the deleterious effects of ovariectomy (Norazlina et al. 2000), orchidectomy (Ima Nirwana et al. 2000), ferric-nitrilo&shy;triacetate (Ahmad et al. 2005) and nico&shy;tine (Hermizi et al. 2007).</p>\r\n<p>\r\n	Our previous study (unpublished) has confirmed that osteoporotic changes were detected histologically as early as four weeks post-ovariectomy in the rat model. In the present study we found that after eight weeks of treatment, ovariec&shy;tomised-rats have increased oxidative stress compared to the sham-operated rats. This was shown by the increase in the lipid peroxidation product, MDA and the decrease in the antioxidant enzyme glutathione peroxidase (GPX). There also appeared to be a decrease in the superoxide dismutase (SOD) levels but the changes were not significant. Among the reactive oxygen species, hydrogen pe&shy;roxide is the most prominent free radicals as it can readily pass through cell mem&shy;branes and cannot be excluded from cells. SOD converts superoxide to hydro&shy;gen peroxide (H<sub>2</sub>O<sub>2</sub>) which is then converted by GPX to water.</p>\r\n<p>\r\n	GPX also degrade H<sub>2</sub>O<sub>2</sub> that originates from the oxidation of polyunsaturated fatty acids (Puglia &amp; Powell 1984). Therefore, the reduction in GPX level would be more significant as it is actively involved in converting H<sub>2</sub>O<sub>2</sub> to water. Muthusami <em>et al.</em> (2005) was able to demonstrate that ovariectomy had re&shy;duced both SOD and GPX activities. However, these anti-oxidant enzymes were measured in the bone, where-as we have measured the SOD level in the plasma. Estrogen deficiency in postme&shy;nopausal women leads to oxidative stress as shown by a decrease in thiol antioxidants, exposing the bone to oxida&shy;tive damage (Lean et al. 2003). Lean <em>et al.</em> (2005) has found that the antioxidants, N-acetyl-cysteine and ascorbate were as effective as estrogen in normalizing the glutathione and thioredoxin reductase activities in rodents in order to prevent ovariectomy-induced bone loss. We have shown in this study that vitamin E, a po&shy;tent antioxidant, has demonstrated a similar protective role against ovariec&shy;tomy-induced oxidative stress by sup&shy;pressing MDA levels and raising GPX and SOD activities. Palm tocotrienols were clearly the better vitamin E in pro&shy;tecting the bones against oxidative dam&shy;age post-ovariectomy. As previously shown, &alpha;-tocotrienol has better anti-oxidant activity than &alpha;-tocopherol (Serbinova et al. 1991, Suzuki et al. 1993). The superior antioxidant activity of tocotrienols may be contributed by its more uniform distribution in the mem&shy;brane lipid bilayer, more efficient interac&shy;tion of the chromanol ring with lipid radi&shy;cals and higher recycling efficiency from chromanoxyl radicals (Serbinova et al. 1991).</p>\r\n<p>\r\n	In the present study, palm-tocotrienols were able to prevent lipid peroxidation in ovariectomised rats. Both &alpha;-tocopherol and palm tocotrienols were found to raise the SOD level with the latter having a more profound effect. While for the GPX level, only palm tocotrienols were able to restore the GPX level of ovariectomised rats back to the sham-operated level. These results have demonstrated the ability of vitamin E to reactivate the anti-oxidant enzymes and guard against insult caused by oxidative stress.</p>\r\n<p>\r\n	Based on our previous study (unpub&shy;lished) that the femur should be osteopo&shy;rotic as early as four weeks post-ova&shy;riectomy, we have assessed the bone strength by running a biomechanical test. In the present study, patterns of reduced bone strength in all the biomechanical parameters were seen in the ovariecto&shy;mised&ndash;control rats. However, even after eight weeks post-ovariectomy, they were not sufficiently low enough to record any significant difference. A study by Xie <em>et al.</em> (Xie et al. 2005), demonstrated signifi&shy;cant reduction in the biomechanical pa&shy;rameters in rats sixteen weeks post-ova&shy;riectomy. Therefore, we conclude that osteoporosis at eight weeks post-ova&shy;riectomy was not severe enough to affect biomechanical properties although os&shy;teoporosis should have set in four weeks earlier. The different strain of rats used in the study by Xie <em>et al.</em> (Xie et al. 2005), which had used Sprague-Dawley com&shy;pared to Wistar in our study may have also accounted for the difference in the findings.</p>\r\n<p>\r\n	In conclusion, palm tocotrienols given as oral supplementation at 60 mg/kg were able to prevent the oxidative stress induced by ovariectomy in a rat model. The efficacy of palm tocotrienols was greater than &alpha;-tocopherol. Palm tocotrie&shy;nols have the potential to be used for the treatment and prevention of post-meno&shy;pausal osteoporosis. A longer post-ova&shy;riectomy period is required to assess the ability of vitamin E in improving the bone biomechanical strength in the post-me&shy;nopausal rat model.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	We would like to thank Mrs Sinar Suriya Muhammad, Mr Muhammad Arizi, Mr Faisal Ariffin and the rest of the Pharma&shy;cology department staff for their technical assistance We would also like to thank Mr. Gapor Md Top from the Palm Oil Re&shy;search Institute of Malaysia (PORIM) for providing the palm oil tocotrienol mixture and the Department of Mechanical Engi&shy;neering and Material for providing the biomechanical test services. The study was supported by a research grant from the Faculty of Medicine, Universiti Kebangsaan Malaysia (FF-226-2006).</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(416, 413, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Vitamin E has been reported to have an&shy;ticancer properties in both animal models and cells in culture (Nesaretnam et al. 1995). The vitamin E usually studied is &alpha;-tocopherol but recently more studies have focused on tocotrienol, isomers of vitamin E present in large quantities in palm oil. The tocotrienols are reported to be stronger antioxidants than &alpha;-tocophe&shy;rol. In addition tocotrienols have proper&shy;ties which are different from &alpha;-tocophe&shy;rol. In this laboratory, GTT have been shown to have antiproliferative effect against a variety of cell lines particularly liver cancer cells with an IC<sub>50 </sub>of 170 &mu;M (Aida et al. 2007).</p>\r\n<p>\r\n	Among the tocotrienol group, delta-to&shy;cotrienol and gamma-tocotrienol are con&shy;sidered to have the strongest inhibitory effects on cancer cell growth (Sakai et al. 2004). Gamma-tocotrienol (GTT) has also been shown to exhibit antiprolifera&shy;tive properties on other tumor cells in culture, such as breast cancer cells (Nesaretnam et al. 1995), murine mela&shy;noma cells (He et al. 1997), human leu&shy;kemia cells (Mo &amp; Elson 1999) and ma&shy;lignant mammary epithelial cells (Shah &amp; Sylvester 2005).</p>\r\n<p>\r\n	Several possible mechanisms of the antiproliferative effect of GTT have been suggested such as its involvement in in&shy;ducing antioxidant effects (Noguchi et al. 2003; Calvisi et al. 2004), suppressive effects on HMG-CoA reductase activity (Parker et al. 1993; Mo &amp; Elson 2004), pro-apoptotic effects (Agarwal et al. 2004; Shun et al. 2004), regulating mito&shy;genesis (Mo &amp; Elson 1999) and anti-an&shy;giogenic potential (Miyazawa et al. 2004). In a recent and more elaborate study, it was shown that GTT inhibits the nuclear factor &kappa;B activation pathway, leading to down-regulation of various gene products and potentiation of apoptosis (Ahn et al. 2007). However, most of these mechan&shy;isms have only been studied on other types of tumor cells in particular breast cancer cell lines and specific pathways have not been fully characterized. To further understand the mechanisms in&shy;volved in the anti-proliferative effects of GTT, it would be of interest to identify proteins whose expression are effected by exposure of HepG2 a liver cancer cell line to GTT treatment by using two-di&shy;mensional gel electrophoresis (2DE).</p>\r\n<p>\r\n	Proteomics has evolved into a robust and highly reliable method allowing re&shy;searchers to profile cellular responses to various physiological and pathological conditions at the protein level. Currently it has been successfully employed in the area of cancer research, particularly in the search for new biomarkers (Cho 2007). Apart from that, it has also been utilized in the analysis of the response of tumor cells to an antitumor substance (Stockwin et al. 2007; Tong et al. 2008; Scheper et al. 2008). The majority of pre&shy;vious studies showed changes in the ex&shy;pression of proteins involved in the cel&shy;lular processes such as apoptosis and proteins that are directly associated with tumor development characteristic of the type of cancer (Yim et al. 2004; Mouat et al. 2005; Li et al. 2006; Tong et al. 2008).</p>\r\n<p>\r\n	In this study, 2DE was used to charac&shy;terize changes in protein expression lev&shy;els in HepG2 cell line following treatment with GTT. By identifying the specific al&shy;terations in the cellular protein profile in response to GTT treatment, the underly&shy;ing mechanisms involved in the anti-pro&shy;liferative activity of GTT may be eluci&shy;dated.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Cell culture and treatment</em></p>\r\n<p>\r\n	HepG2 and WRL68 cell lines were pur&shy;chased from American Type Culture Collection (ATCC, USA). Cells were maintained in Eagle minimum essential medium (EMEM, Flowlab, Australia) containing 10% foetal calf serum (PAA, Austria), penicillin/ streptomycin (100 &mu;g/ml) (Flowlab, Australia) at 37&deg;C in an atmosphere containing 5% CO<sub>2</sub>. Gamma-tocotrienol (GTT) was supplied by Malaysian Palm Oil Board (MPOB). Stock solution of GTT (0.5 M) was pre&shy;pared in 100% ethanol and stored as small aliquots at -20&ordm;C. Prior to use, GTT from stock solution was mixed with foetal calf serum and incubated overnight at 37&ordm;C. Culture medium and 100% ethanol were then added to give a final concen&shy;tration of 70 &mu;M. When the cells reached confluency, they were trypsinized, cen&shy;trifuged and counted using a haemocy&shy;tometer. Cells were plated at a density of 2 x10<sup>6</sup> cells/100-mm culture plates for 24 hrs and incubated with 10 mL of 70 &mu;M GTT enriched medium for 48 hours. As a negative control, cells were cultured in EMEM without GTT.</p>\r\n<p>\r\n	<em>Sample Preparation for 2DE</em></p>\r\n<p>\r\n	At the end of the treatment period, cells were harvested by trypsinization and transferred into 15 ml Falcon centrifuge tubes. The cells were harvested by cen&shy;trifugation at 800 rpm for 10 minutes. They were then washed thrice with cold PBS and the resulting pellet resuspended in 200 &mu;l lysis buffer (8M urea, 2% CHAPS, 0.5% pH 3-10 IPG buffer, Amersham, USA) containing protease inhibitor mix (Amersham, USA). The cells were then incubated on ice for 30 min&shy;utes with intermittent vortexing at 10 minute intervals. After centrifugation at 13000 rpm for 30 minutes at 4<sup>o</sup>C, the su&shy;pernatant was transferred to sterile mi&shy;crocentrifuge tubes. Protein concentra&shy;tion was determined as described by Bradford (1976).</p>\r\n<p>\r\n	<em>Two-dimensional Gel Electrophoresis</em></p>\r\n<p>\r\n	First dimension separation was carried out on immobilized pH gradient strips (24 cm, linear, pH 3-10, GE Healthcare) with Ettan IPGphor II Isoelectric Focusing System and standard strip holder (GE Healthcare). Isoelectric focusing (IEF) was performed under the following con&shy;ditions: 500V for 1 hour, 1000V for 1 hour, 8000V for 3 hours and finally 8000V for 3 to 4 hours. Protein samples (60 &mu;g) were loaded into sample cups at the anode end. Upon completion of IEF, strips were equilibrated in equilibration buffer (6M urea, 75 mM Tris-HCl, pH 8.8, 29.3% Glycerol, 2% SDS, 0.002% bro&shy;mophenol blue, 1% DTT) for 15 minutes, followed by the same buffer containing 25% iodoacetamide instead of DTT for another 15 minutes. The second dimen&shy;sion separation was carried out at 15<sup>o</sup>C on 12.5% SDS slab gels using an ETTAN DALT II electrophoresis system (GE Healthcare), with the IPG strips sealed on the top of the gels with 0.5% agarose. SDS-PAGE was run at constant power of 1 W/gel for 1 hour, then switched to 13 W/gel until the bromophenol blue marker reached the bottom of the gel. For every treatment group, triplicate runs were made to ensure the accuracy of subse&shy;quent analysis.</p>\r\n<p>\r\n	<em>Gel Staining and Image Analysis</em></p>\r\n<p>\r\n	Protein spots were visualized by silver staining as described in the PlusOne Sil&shy;ver Staining Kit (GE Healthcare). Images of stained 2DE-gels were acquired with a UMAX scanner, model UTA-2100 XL and stored as TIFF images. The 2DE maps were then analyzed using the 2D Image Master Platinum software Version 6.0 (GE Healthcare). The volume of each spot in a gel was normalized as a per&shy;centage of the total volume of all spots detected on the gel. Only those spots that were consistently changed (more than 2-fold) were selected as spots of interest in this study.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The proteome profile obtained from the analysis was highly reproducible between different runs and different sample sets. Representative gels are shown in Figure 1 and Figure 2. A total of ~500 individual protein spots were detected with silver staining. Following image analysis, a total of 83 spots were identified as being dif&shy;ferentially expressed following GTT treatment. Out of the 83 spots, only 18 individual protein spots were differentially expressed in a consistent manner in every triplicate gel, and therefore, were selected as spots of interest in this study. These spots are shown in Figure 3.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	In general, the 2DE gels were highly re&shy;producible in terms of spot number, spot intensity and general protein profile. There were minimal intra and inter-sam&shy;ple variation among the 2DE gels. Con&shy;sistency and reproducibility were achieved by standardized procedures of sample preparations of cells cultured, conditions for first and second dimen&shy;sions of electrophoresis, gel staining and image acquisition. During sample prepa&shy;ration, a protease inhibitor cocktail was used. With the use of this inhibitor cock&shy;tail, resulting 2DE gels did not show any signs of protein degradation due to pro&shy;tease activity (as evidenced by the high resolution of high molecular weight pro&shy;teins).</p>\r\n<p>\r\n	Twenty four centimeter gels were used in order to obtain the best possible reso&shy;lution for 2DE. A sample loading of 80&mu;g protein per gel resulted in 2DE profiles with saturated protein spots which may lead to inaccuracies during quantitative analysis. A representative gel of an 80&mu;g protein load is shown in Figure 4. To re&shy;solve this problem, a sample loading of 60&mu;g protein per gel was used. This was found to be ideal for resolving the highest number of spots without sacrificing gel resolution. Image analysis revealed that there were no saturated spots present at this protein loading therefore ensuring that image analysis would remain quan&shy;titative. Thus at this point, we have es&shy;tablished a robust and reliable protocol for the 2DE analysis of the proteome of HepG2 cell lines. We then moved on and performed differential proteomics analy&shy;sis of treated and untreated HepG2 cell lines.</p>\r\n<p>\r\n	A total of 18 differentially expressed protein spots were identified following treatment of HepG2 cells with GTT. Among the protein spots, 7 of them (spot no 1, 2, 3, 4, 5, 6 and 18) are acidic (pI &lt; 5) proteins. Of these seven, protein spot numbered 1 has the largest molecular weight (&gt;90 kDa), while protein spot numbered 18 is the smallest sized pro&shy;tein with molecular weight around 10 kDa. The rest (spot no 2, 3, 4, 5 and 6) are medium-sized proteins with a mo&shy;lecular weight ranging from 30 to 50 kDa. A group of 8 protein spots (spot no 8, 9, 10, 11, 12, 13, 14 and 15) are found ac&shy;cumulated in the middle of the 2D-PAGE gel of both group of HepG2 cells (with and without GTT treatment). All of these proteins have a molecular weight ranging from 35 to 60 kDa. The pI of these pro&shy;teins could vary from slightly acidic, neu&shy;tral, to slightly basic. This cannot be con&shy;firmed because all the proteins were too close between each other. Another two (spots no 7 and 16) are assumed as slightly basic proteins since they were both well-separated on the slightly basic region of the 2D-PAGE gel. These spots were large proteins with the molecular weight around 60 to 90 kDa. The last protein from the 18 differentially ex&shy;pressed proteins were highly basic pro&shy;teins with spot numbered 17 as average-sized protein (around 30 kDa). These observations showed that the changes in protein expression occur over a wide pH and molecular weight range and were not localized to any specific region of the gel. This was expected as treatment with GTT will most likely affect a wide variety of proteins.</p>\r\n<p>\r\n	Interestingly, all of these 18 protein spots were observed only in HepG2 cells without GTT treatment and not in the HepG2 cells with the GTT treatment. In other words, these proteins were greatly decreased to the extent that they were no longer detectable in the 2D protein profile of HepG2 cells with GTT treatment. Whether these proteins are directly re&shy;lated to the antiproliferative activity of GTT remains unclear as we have yet to identify these proteins.</p>\r\n<p>\r\n	Our results demonstrated that GTT treatment caused specific and significant changes in the 2DE profile of the HepG2 cell line. Proteins generally function in groups that are involved in many biologi&shy;cal pathways simultaneously. Thus, any interventions will ultimately affect the regulation and/or modulation of not only one but many proteins simultaneously. This has been continuously demon&shy;strated in various studies of the intracel&shy;lular mechanisms of various drugs. One example is the study conducted by Ahn et al. (2007), demonstrated that GTT sup&shy;pressed NF-&kappa;B activation pathway and NF-&kappa;B-regulated gene products. As in our study, the unknown proteins that are involved might be associated with the anti-tumor activity of GTT in HepG2 cells. Thus, further identification of these pro&shy;teins by mass spectrometry may lead to a greater understanding of the mecha&shy;nisms involved in the anti-proliferative effects of GTT.</p>\r\n', 1),
(417, 414, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Chemoprevention is a promising strategy to prevent cancer by using natural or synthetic substance. Many studies have successfully shown the effectiveness of diets having chemopreventive effects against the growth of cancer cells. White and green tea ameliorated colon cancer by inducing apoptosis and modulation of certain genes (Santana-Rios et al. 2002, Orner et al. 2003). Curcumin in tumeric induced apoptosis by activating caspases in malignant glioma cells (Gao et al. 2005). <em>Zingiber officinale</em> or commonly called ginger is widely used for medicinal purposes since thousands of years ago in Chinese, Arabs, Romans and Indian medicine (Ross 2002). In our previous study, ginger extract was shown to re&shy;duce lipid peroxidation and tumour bur&shy;den in hepatocarcinogenesis-induced rats. Other studies had reported that gin&shy;ger exerted its anti-tumour properties by inhibiting proliferation and inducing apoptosis in HL-60 leukemia cells (Surh &amp; Lee 1998) and Jurkat human T cell leukemia cells through down-regulation of anti-apoptotic protein Bcl-2 and en&shy;hancement of pro-apoptotic protein Bax<br clear="all" />\r\n		(Miyoshi et al. 2003).</p>\r\n	<p>\r\n		Cancer is caused by an imbalance in the rate of proliferation and apoptosis or cell death. Apoptosis is a form of programmed cell death characterized by morphological changes in cells executed by cysteine-aspartate proteases (cas-pases) and regulated by the Bcl-2 family of proteins involved in the signal trans-duction pathways (Coultas &amp; Strasser 2003, Hanson <em>et al</em>. 2008). It is the preferential way of targetting and removing cancer cells. Dietary com-pounds that can trigger apoptosis would be a potential use in cancer chemo-prevention.</p>\r\n	<p>\r\n		Hepatocellular carcinoma (HCC) is the fifth most common cancer worldwide with nearly half a million of new cases an&shy;nually (Wei 2006). In Malaysia, HCC is the eleventh most common cancer among all malignant diseases (Gerard &amp; Halimah 2003). The attributable risk fac&shy;tors of HCC are chronic hepatitis B and C infections, cirrhosis, exposure to aflatoxin and certain chemicals such as polyamine hydrocarbon (PAH), diethylnitrosamine (DEN) and 2-acetylaminofluoren (AAF) (Schafer &amp; Sorrell 1999). Currently, the treatments of HCC include surgical re&shy;section, liver transplantation, drug and radiation therapy (Forner et al 2006). All these procedures posed a lot of risk and side effects. The search for an alternative and safe compound or chemopreventive agent would benefit patients and reduce the unwanted side effects and risk fac&shy;tors. Ginger is an indispensable ingre&shy;dient of Asian foods and this report con&shy;cerns the chemopreventive efficacy of ginger in an ethionine induced liver can&shy;cer carcinogenesis model by examining the expression of pro- and anti-apoptotic proteins, caspase-8 and Bcl-2 respec&shy;tively.</p>\r\n	<p>\r\n		<strong>MATERIALS AND METHODS</strong></p>\r\n	<p>\r\n		<em>Animals, Chemicals and Treatment</em></p>\r\n	<p>\r\n		Thirty male Wistar albino rats aged be&shy;tween 3-4 months and weighing 200-250 g were supplied by the Animal Care Unit of Universiti Kebangsaan Malaysia (UKM) Medical Center. The study was approved by the Animal Ethics of the Fa&shy;culty of Medicine, UKM. Animals were kept in a polycarbonate cage provided with food and water<em>ad libitum</em>. They were maintained under standard condi&shy;tions of temperature and humidity with an alternating light and dark cycle. Rats were randomized into 5 groups of 6 ani&shy;mals each. The first group and the second group served as the control group and were fed with normal rat chow (Gold Coin, Malaysia) and rat chow plus olive oil respectively. The latter served as a control for gavage method and the deli&shy;very of ginger. Rats in group 3 received ginger extract at 100 mg/kg body weight by gavage method. Ginger extract was prepared by ethanol extraction and kept at 4<sup>0</sup>C. It was dissolved in olive oil and force-fed to the rats. Rats in group 4 were fed with choline deficient diet (ICN Biochemicals, USA) plus 0.1% ethionine (Sigma Chemical Co., USA) in drinking water, known as CDE. This is the model to induce the production of oval cells, which are the precursor cells of liver can&shy;cer(Akhurst et al. 2001). Rats in group 5 received ginger as in group 3 plus CDE diet. All rats were killed at 8 weeks and the liver tissues were excised after perfu&shy;sion and embedded in paraffin blocks for immunohistochemical staining.</p>\r\n	<p>\r\n		<em>Liver perfusion and preparation of paraf&shy;fin blocks</em></p>\r\n	<p>\r\n		The rats were sacrificed using ether. All equipments for dissection were sterilized using 70% alcohol before use. The rats were anasthesized intraperitoneally with Zoletil 50 (0.1 ml/100g body weight), fol&shy;lowed by heparin (25, 000 U/ml) injection to the inferior vena cava. The portal vein was then canulated using an intravenous catheter, size 16G (2.25 inches) for the perfusion procedure. The liver was then perfused in PBS, pH 7.4 for 1 minute at a 10ml/min flow rate at room temperature, followed by 1:1 ratio of 4% para-formal&shy;dehyde and 0.1% glutaraldehyde for 3 minutes. Then the liver was perfused back in PBS for another 2 minutes. A portion of the perfused liver was then immersed in 10% formalin for fixation before embedding in paraffin.</p>\r\n	<p>\r\n		<em>Preparation of tissue sections</em></p>\r\n	<p>\r\n		The paraffin-embedded tissues were cut at 3 &mu;m thick with a rotatory microtome(Leica, Germany). The tissue sections were placed on poly-L-Lysine (Sigma-Aldrich Co. USA) treated slides with 1:10 dilution. The slides were then dried overnight and stored at room tempe-rature until used for staining.</p>\r\n	<p>\r\n		<em>Hematoxylin &amp; eosin (H&amp;E) staining </em></p>\r\n	<p>\r\n		The sections were deparaffinized and hydrated with sequential washes in xy&shy;lene and alcohol. Nuclei were stained by immersing in Mayer&rsquo;s hematoxylin solu-tion (Lab Vision Corp., UK) for 8 mi&shy;nutes and rinsed under running tap wa&shy;ter. The slides were then dipped in 1% acid alcohol to remove excess hematox&shy;ylin followed by immersion in 2% sodium acetate. Slides were rinsed in running water followed by eosin staining for 5 min to stain the cytoplasm. Finally, slides were dehydrated through a series of graded alcohols and mounted withdibutylpthalate xylene (DPX).</p>\r\n	<p>\r\n		<em>Immunohistochemistry for detection of Bcl-2 and caspase-8</em></p>\r\n	<p>\r\n		Paraffin sections (3 mm thick) were cut from liver specimens. Sections were de&shy;paraffinized and rehydrated by sequential immersion in xylene, a series of alcohol concentrations (100, 95, 80 and 70%), and in running water. Slides were pre-incubated in 3% hydrogen peroxide for 10 minutes to block endogenous perox&shy;idase activity. Slides were washed in Tris-HCl-buffered saline (TBS) before incubating with bovine serum albumin and biotin as a blocking step to reduce nonspecific staining. For detection of Bcl protein, slides were then immersed in target retrieval solution (pH9.9) (DAKO, U.S.A.) at 98<sup>o</sup>C in a water bath for 20 minutes. For caspase-8, the target re&shy;triev&shy;al solution (pH9.9) (DAKO, U.S.A.) was heated in microwave oven for 20 minutes at 98<sup>o</sup>C. Slides were left at room temperature for 20 minutes before washing in Tris-buffered saline (TBS) for 3 changes at 3 minutes each. Tissues were then incubated with the primary an&shy;tibody. For evaluation of Bcl-2 expres&shy;sion, tissues were incubated with mono&shy;clonal mouse anti-human Bcl-2 (DAKO, Denmark) at 1:50 dilution for 30 minutes while for the evaluation of caspase-8, tissues were incubated with rabbit poly&shy;clonal to human Caspase-8 (Abcam, UK) at 1:100 dilution for 1 hour.&nbsp; After several washings, slides were then incubated with secondary antibody conjugated with biotin and streptavidin labeled with horse&shy;radish peroxidase (LSAB kit, DAKO, Denmark) for 30 minutes. The slides were then treated with diamino-benzi&shy;dine, DAB (DAKO, U.S.A.) for 20 minutes before counterstaining with haematoxylin for visualization of antigen. TBS was used in place of the primary antibody for the negative control. Human tonsil and gastric tissues were used as positive controls for Bcl-2 and caspase-8 respec&shy;tively.</p>\r\n	<p>\r\n		<em>Immunostaining analysis </em></p>\r\n	<p>\r\n		Immunoreactivity evaluation was based on staining intensity and percentage of positive staining of Bcl-2 and caspase-8. Staining intensity was divided into 3 cate&shy;gories: 3+ indicates strongly positive, 2+ indicates moderately positive, and 1+ indicates slightly positive. The percen&shy;tage of positive staining was determined from 1% to 100% of cells stained posi&shy;tively at 10 different fields observed un&shy;der 400X magnification of light micro&shy;scope. Twelve slides (2 slides from each rat) were prepared from a total of six rats to represent each group.</p>\r\n	<p>\r\n		<em>Statistical analysis</em></p>\r\n	<p>\r\n		Descriptive analysis was used to com&shy;pare the expression of apoptotic protein in different sample groups.</p>\r\n	<p>\r\n		<strong>RESULTS</strong></p>\r\n	<p>\r\n		<em>H&amp;E staining for oval cells expression</em></p>\r\n	<p>\r\n		The control groups (normal rat chow, normal rat chow + olive oil) and rats treated with ginger showed normal rat liver histology with hexagon shaped he&shy;patocytes, round nucleus and distinct cytoplasm. The sinusoids were clearly visible under 400x magnifications with Kupffer cells located at the sinusoidal wall (Figure 1A). A similar morphology of liver cells was also observed in the gin&shy;ger extract group (Figure 1C) and CDE group treated with ginger extract (Figure 1E). Abnormal liver cell morphology was observed in the liver-cancer induced group with irregular shaped hepatocytes and sinusoids (Figure 1D). Numerous oval-shaped cells with large nucleus and scanty cytoplasm located mainly near the periportal site were seen in the liver-can&shy;cer induced group (CDE).</p>\r\n	<p>\r\n		Table 1 shows increased oval cell ex&shy;pression in CDE group (91.6% of sam&shy;ple) which was abrogated after treatment with ginger extract (100 mg/kg body wt).</p>\r\n	<p>\r\n		<em>Bcl-2 expression</em></p>\r\n	<p>\r\n		Figure 2 represents patterns of Bcl-2 ex&shy;pression in control groups and in CDE group treated with ginger. Figure 2A shows positive staining for Bcl-2 protein in tonsil tissue.</p>\r\n	<p>\r\n		There were no Bcl-2 expression ob&shy;served in the control (and olive oil group, image not included) and ginger extract groups (Figures 2B and 2C). However, Bcl-2 expression was clearly observed in the CDE group (Figure 2D) with 91.6% of the cells positively stained (Table 2) with the following intensities: 66.6% cells with 1+ intensity and 25% cells with 2+ inten&shy;sity (Table 3). Oval cells that were stained with Bcl-2 showed patchy ex&shy;pression in the cytoplasm with no ex&shy;pression observed in the nucleus (Figure 2D, 400x). Treatment of ginger inhibited the expression of Bcl-2 in the CDE group (Figure 2E).</p>\r\n	<p>\r\n		<em>Caspase-8 expression</em></p>\r\n	<p>\r\n		Figure 3 represents patterns of caspase-8 expression in control and ginger groups and in CDE group treated with ginger. Figure 3A shows positive staining for caspase-8 in human gastric tissue. The expression of caspase-8 was observed in both CDE and CDE group treated with ginger extract (Figure 3D, 3E). No cas&shy;pase-8 expression was observed in the control and ginger groups (Figures 3B, 3C). For CDE group, 41.7% of cells were stained with caspase-8 (Table 4) with the following intensities: 33.4% cells with 1+ intensity and 8.3% cells with 2+ intensity (Table 5). However, for CDE group treated with ginger extract, 100% of the cells were stained positively for caspase-8 with the following intensities: 50% with 3+ intensity, 25% with 2+ inten&shy;sity and 25% with 1+ intensity (Table 4). Cas&shy;pase-8 was expressed in a patchy pat&shy;tern in the cytoplasm of the oval cells of CDE group (Figure 3D). A homogen&shy;ous staining of caspase 8 was observed in the CDE group treated with ginger ex&shy;tract (Figure 3E).</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		Most of the flavouring ingredients in Asian foods are rich in phytochemicals with medicinal properties which include turmeric, cloves, garlic, aniseed, mus&shy;tard, saffron, cardamom and ginger (Sengupta et al. 2004). Some of these spice condiments have been supported by experimental models to have chemo&shy;preventive properties which have the ability to interfere with carcinogenic process. The purpose of chemopreven&shy;tive agent in cancer prevention is to block or cause delay in onset of cancer, pro&shy;gression from precancerous lesion or recurrence after treatment (Tsuda et al. 2004).&nbsp;&nbsp;</p>\r\n	<p>\r\n		Ginger has been used in time of im&shy;memorial for food condiments and medi&shy;cinal use especially to aid digestion and treat stomach upset, diarrhoea and nau&shy;sea. It was shown to have strong anti-oxidant properties attributed by the bio-active components such as 6-paradol, shogaol, zingerone and gingerol (Masuda et al. 1995, Aeschbach et al. 1994, Chang et al. 1994, Cao et al. 1993, Reddy &amp; Lokesh 1992).&nbsp; Antioxidative capacity of ginger has been associated with the ability of ginger to inhibit carci&shy;nogenesis by reducing oxidative stress and inducing apoptosis (Shukla &amp; Singh 2006, Katiyar et al. 1996, Manju &amp; Nalini 2005, Rhode et al. 2007).</p>\r\n	<p>\r\n		Our study clearly showed the efficacy of ginger having antitumour properties by inhibiting the proliferation of oval cells as evidenced by the reduced number of oval cells and the perpetuation of normal his&shy;tological structure of liver tissues in liver cancer induced rats treated with ginger extract. Ovalcell proliferation precedes neoplasia in many rodent modelsof he&shy;patocellular carcinoma and in chronic liver disease of human studies (Ackhurst et al. 2001, Lowes et al. 1999). Preven&shy;tion of this proliferativeresponse can re&shy;duce the risk of subsequent carcinoma. This was supported by our previous findings which showed that ginger re&shy;duced oval cell proliferation and liver tu&shy;mour formation in hepatocarcinogenesis induced rats (Mohd Habib et al. 2008). This property of ginger could be attri&shy;buted to the presence of high phenolic compounds such as [6]-gingerol, and [6]-paradol. [6]-gingerol has been shown to suppress experimental metastases in tumour-bearing mice skin carcinogenesis probably via its anti-angiogenic activity (Kim et al. 2005) and inhibition of COX-2 expression along with suppressed NF-kB DNA binding activity (Kim et al. 2004). Since tumour promotion is closely linked to oxidative stress, a compound that ex&shy;hibits antioxidant properties could act as an anticarcinogenic agent (Shukla &amp; Singh 2006).&nbsp;</p>\r\n	<p>\r\n		Some compounds present in ginger may exert cancer preventive effects by inducing apoptosis in cancerous or transformed cells. (Shukla &amp; Singh 2006). Several studies have reported that compounds in ginger suppress prolifera&shy;tion of human cancer cells through in&shy;duction of apoptosis accompanied by downregulation of anti-apoptotic protein Bcl-2 and enhancement of pro-apoptotic protein Bax expression (Lee &amp; Surh 1998, Miyoshi et al. 2003).&nbsp; The dimi&shy;nished expression of anti-apoptotic Bcl-2 protein observed in CDE group treated with ginger correlated with inhibition of oval cell expression. This confirmed that ginger must have bioactive compounds to produce such chemopreventive ef&shy;fects. Our findings are supported by Reed et al. (1994), who have shown that Bcl-2 prevented the death of neoplastic cells via apoptosis. Vaux et al. (1988) reported that expression of Bcl-2 induced the tumour formation in mice. Our study showed patchy staining of Bcl-2 in oval cells and the results are supported by Frommel et al. (2000) who reported that Bcl-2 was only expressed in proliferative cells near the periportal site of liver (oval cells) but not in normal hepatocytes, Kupffer cells and bile duct epithelium.</p>\r\n	<p>\r\n		Caspase-8 is an executor zymogen protein involved in apoptosis signaling pathway. Ishiguro et al (2007) proved that [6]-gingerol facilitated TRAIL-induced apoptosis by activation of caspase-3/7 activity in human gasric cancer cells. We observed caspase-8 staining in both CDE and CDE group treated with ginger, with enhanced staining observed in the latter. This observation confirmed that in cancer cells, apoptosis is a natural way of inhi&shy;biting its growth, and chemopreventive agents, having the ability of inducing apoptosis, enhance further the effect of apoptosis as seen with the effect of gin&shy;ger extract.</p>\r\n	<p>\r\n		In conclusion, our study agrees with other studies which provide substantial evidence that ginger extract are effective inhibitors of carcinogenic process exhi&shy;bited by reduction of oval cells, Bcl-2 protein and induction of caspase-8 ex&shy;pression.</p>\r\n	<p>\r\n		&nbsp;</p>\r\n</div>\r\n', 1),
(418, 415, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Learning environment in any institutions plays an important role in producing highly competent graduates. In the nursing programme, the main core busi&shy;ness is to produce nursing graduates who can render comprehensive care and treatment to the community. Thus, the most important aspects are the teaching activities and student-teacher interaction in daily environment in the university. In the learning environment, teachers have a great influence on effective learning. This is because the students&rsquo; achieve&shy;ment or outcomes in the programme is dependent on how ef&shy;fective the teacher delivers his or her ex&shy;pertise knowledge in the class room and clinical areas.&nbsp;</p>\r\n	<p>\r\n		Gordon et al. (2000) pointed out that a teacher who describes the learning envi&shy;ronment and the benefits to be gained from a particular patient in a ward or clinic can help students to focus their at&shy;tention on those benefits. Similarly, a teacher who organizes particular tasks or roles for a student or small group ahead of time can make maximal use of what&shy;ever the environment can offer.</p>\r\n	<p>\r\n		In the nursing profession, the teacher is the one who knows nursing education, the aims set for each practice, as well as the student nurses&rsquo; skills. The teacher should be able to point out the important aspects concerning the curriculum. In the clinical setting environment, even though the clinical instructor knows the ward on which the student is practicing, the teacher is still more capable than the clinical instructor in pointing out things that are important for each particular practice (Papp et al., 2003). Therefore in the nursing program integration between the clinical practice setting and school should be developed further to establish the best learning environment for the students. Collaboration between teachers and clinical instructors is necessary to ensure appropriate learning experiences for students according to their individual needs (Newton &amp; Smith, 1998).</p>\r\n	<p>\r\n		If a teacher or clinical instructor does not give the students enough support regarding the planning and implementa&shy;tion of practical nursing situations, clinical learning is experienced as difficult (Nolan, 1998). Therefore, the teacher and clinical instructor are the most po&shy;werful variables in the learning environ&shy;ment (Hutchinson, 2003). We can con&shy;clude that teacher&rsquo;s and clinical instruc&shy;tor&rsquo;s actions, attitudes, enthusiasm, inter&shy;est and expertise will affect their stu&shy;dents&rsquo; learning process directly or in&shy;directly.&nbsp;</p>\r\n	<p>\r\n		The study aims to investigate the IIUM nursing students experience among the various years towards their teachers and to identify the relationship between teaching and students learning percep&shy;tion in their learning environment.</p>\r\n	<p>\r\n		<strong>MATERIALS AND METHODS</strong></p>\r\n	<p>\r\n		This study used quantitative method. It was a cross &ndash; sectional study using questionnaires through a survey. The Dundee Ready Education Measure (DREEM) is an internationally validated, non-culturally specific inventory that pro&shy;vides medical and health profession teachers with a diagnostic tool to mea-sure the state of their school&rsquo;s learning and teaching climate (Pimparyon et al., 2000). It could produce global reading and diagnostic analysis of undergraduate learning environment in medical schools and other health profession institutes.&nbsp;</p>\r\n	<p>\r\n		This study was utilized two out of five subscales in DREEM. The subscales used were students&rsquo; perception of learn&shy;ing (SPoL) and students&rsquo; perception of teacher (SPoT) and total items in these both subscales are 12 and 11 items re&shy;spectively. Items on the DREEM inven&shy;tory scored 4 for Strongly Agree (SA), 3 for Agree (A), 2 for Uncertain (U), 1 for Strongly Disagree (SD). For negative statements reverse coding was required.</p>\r\n	<p>\r\n		<strong>RESULTS</strong></p>\r\n	<p>\r\n		The respondent rate among the IIUM nursing students was 98.1%, which is 105 out of 107 students returned the complete surveys form. All the respon&shy;dents underwent matriculation at IIUM foundation centre prior to enrolling in the nursing bachelor program and were staying together in the same hostel ac&shy;commodation provided by IIUM on cam&shy;pus zone. The questionnaire results re&shy;vealed that IIUM nursing students scored 28.54/48.00 in (SPoL) and 28.13/44.00 in (SPoT). Both findings suggested the IIUM nursing students&rsquo; experience their teachers and the learning are moving towards in positive directions. The results of regression analysis, in which the rela&shy;tionship on perception of learning and teacher were investigated and 51% of the total variation in students&rsquo; perception of teacher score is explained by students&rsquo; perception of learning.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		In the present study, the both total score subscales are very similar with previous studies. Zamzuri et al. (2004), Bassaw et al. (2003) and Whittle et al. (2007) re&shy;ported scores of 28.47, 27.97 and 27.50 respectively. Currently, there is only one previous study which reported a higher score (34.57) in subscales (SPoL). This was a UK study in a fully implemented student &ndash; centered medical school (Miles &amp; Leinster, 2007). Although the overall subscales (SPoL) score in the current study&nbsp; falls in the category of a more positive perception, 2 out of 12 items were poorly scored by the IIUM nursing students (mean score of less than two). These are item 13 <em>(the teach&shy;ing is stu&shy;dent centered</em>) and item 25 (<em>the teaching over emphasis factual learning</em>) (Table 1).</p>\r\n	<p>\r\n		The researchers strongly agree that listening to the expression of students is an important consideration for an educa&shy;tional institution. Genn (2001) suggested a shift to a more student centered rather than the current teacher centered ap&shy;proach should be implemented in current higher education learning institutions. Consideration must also be given to a collegiate, cooperative staff &ndash; student relationship and a cooperative rather than competitive student-student interac&shy;tion.</p>\r\n	<p>\r\n		&nbsp;The overall mean score for (SPoT) generally suggests that the students per&shy;ceived their teachers as moving in the right direction. Intan (2007), in a local DREEM study, reported a higher mean score of 31.94 for this subscale. In con&shy;trast, two others studies from the UK, ie. Miles &amp; Leister (2007) and Whittle et al. (2007) showed scores of 31.30 and 30.40, respectively. The researchers re&shy;ported that the students provided higher overall mean score although there were still items which obtained a mean score of less then 2.00. Thus, it is difficult to perceive a perfect teacher from the stu&shy;dent&rsquo;s viewpoint.</p>\r\n	<p>\r\n		In this present IIUM study, one item showed a mean score of less then 2.00 ie. item number 9 <em>(the teachers are au&shy;thoritarian).</em> The researchers assume that the bachelor of nursing students said this about their teachers because the kulliy&shy;yah has not really focused on a student centered curriculum yet. This finding is similar to other DREEM studies done in the Middle East (Awdah et al., 2004) and Trinidad (Bassaw et al., 2003).&nbsp; Both these studies reported on institutions which use teacher centered curricula where students also score less than 2.00 for the same item.&nbsp;</p>\r\n	<p>\r\n		It is important to know students&rsquo; un&shy;derstanding of the term &ldquo;authoritarian&rdquo; and the characteristics of such an au&shy;thoritarian teacher. This will allow us to assess their acceptability of a teacher who has these characteristics. Interven&shy;tions can then take place to improve the situation in the future. When students have to follow everything that the teacher orders, it results in the creation and moulding of a &lsquo;timid student&rsquo; rather than producing a high spirited and well-moti&shy;vated student. During this level of higher educational, the students should be en&shy;couraged to study based on their interest and self &ndash; actualization rather than to study just because they are afraid of their teachers.</p>\r\n	<p>\r\n		As a result, these two subscales most probably should reflect the same out&shy;come such as in their academic perform&shy;ance and experience greatly during their student life on campus. The arising is&shy;sues from this DREEM study at IIUM embrace the need for the creation of supportive environment as well as de&shy;signing and implementing interventions to remedy unsatisfactorily elements of the learning environment for more effec&shy;tive and successful teaching and learning to be realised.</p>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(419, 416, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Cardiovascular disease is the third lead&shy;ing cause of death in Malaysia, and ac&shy;counted for 29% deaths in 2002 (Ministry of Health, 2004). By international stan&shy;dards, this figure is of concern for Malay&shy;sia. Despite much scientific research and technological advancements, coronary heart disease remains a major cause of premature morbidity and mortality. Pa&shy;tient education has been considered as an integral component of care for patient after myocardial infarction (MI). Following an acute MI, the patient requires infor&shy;mation and knowledge related to their conditions to reduce anxiety and aid re&shy;covery (Almo-Roijer et al. 2004). With information related to MI, patient and family members are able to make suita&shy;ble changes in their lifestyle. Such un&shy;dertaking is easier to be made if a patient understands the basic rationale for changes and the benefits.</p>\r\n<p>\r\n	Although teaching has been an integral component of nursing practice, in gen&shy;eral, nurses are incompetent and lack confidence to deliver an effective patient education to post MI patients. Nurses play an important role to reemphasize information related to myocardial infarc&shy;tion such as life style, medications, diet, psychological concerns, activities, stress management and symptoms. All these are crucial so that patient can manage the post discharge period safely and make informed decisions about potential lifestyle changes (Timmins 2005; Burney et al. 2002).</p>\r\n<p>\r\n	Nurses have preconceived ideas about what information is required for post MI patients which may cause them to over&shy;look the genuine information needed by the patients. Hence, it is realistic to con&shy;clude that some of the important priorities of education process were overlooked. Knowles Theory of Andragogy, (1989), an adult learning theory, provides a guide by the principle that people tend to learn what they perceive as important to them. It is unlikely that the informal educational approach which is mostly unplanned and haphazard in manner will be ineffective in meeting the information needs of post MI patient. To achieve optimum patient&rsquo;s health education objective, the nurses should possess the ability to identify the post MI patient&rsquo;s own perception and readiness to learn and acceptance of the information of what are relevant at this point of time (Timmins &amp; Kaliszer 2002).</p>\r\n<p>\r\n	In addition, Chan et al. (2004) stated that early commencement of patient edu&shy;cation enhances quality of life and posi&shy;tive outcome to the post MI patients. As the post MI patients&rsquo; information needs depend upon nurses to get their needs met, it is crucial to conduct this study in UKMMC. The objective of this study was to identify nurses&rsquo; perception on informa&shy;tion needs of post MI patients of UKMMC. As there is a dearth of research on this topic, the results of this study should be able to highlight the impor&shy;tance of information needs among post MI patient of UKMMC in a meaningful way that will contribute to improving the overall delivery of quality nursing care.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross sectional study using Cardiac Patient Learning Needs Inventory (CPLNI) adopted from Timmins &amp; Kalizer (2002) was used. The CPLNI was devel&shy;oped by Gerard (1982) to measure the congruency in perception of learning needs between the registered nurse and post myocardial infarction patients. The CPLNI contained 27 items which were arranged into seven categories: related to anatomy and physiology, psychologi&shy;cal factor, life style factor, medication information, dietary information, physical activity, and symptom management. Nurses&rsquo; socio-demographic data con&shy;sisted of age, ward placement, length of services and marital status. This study was conducted at Coronary Care Unit (CCU), Coronary Rehabilitation Ward (CRW), Medical Ward 1 and Medical Ward 2 of UKMMC from January 2007 to March 2007. An illustration of the attrib&shy;utes of this study, as a conceptual framework is shown in figure 1.</p>\r\n<p>\r\n	At the time of this proposed study, there were no formal classes offered to cardiac patients at UKMMC. Individual&shy;ized instruction was given to patients by registered nurses without standard proto&shy;col. A sample of 62 registered nurses was asked to complete the questionnaire. These registered nurses have had vary&shy;ing educational backgrounds and levels of experience teaching myocardial infarc&shy;tion patients. Nurses who worked less than a year in the CCU, CRW, Medical Ward 1 and Medical Ward 2 and not con&shy;sented to participate in the study were excluded.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Respondents of this study which con&shy;sisted of 56 nurses (90%) were recruited. Six nurses (10%) did not complete the ques&shy;tionnaire given. A descriptive data of the respondents were presented in table 1. The findings of this study reported that CCU and CRW nurses ranked symptom management as top priority followed by medication and lifestyle factor. However, nurses in Medical Ward 1 and Medical Ward 2, ranked medication, symptom management and psychology factor as the top priority (Table 2).</p>\r\n<p>\r\n	There were significant differences be&shy;tween ward nurses&rsquo; responses with their perception related to anatomy and physi&shy;ology, psychology factor, life style factor, physical activity, and symptom manage&shy;ment with a p value&lt;0.05 (Table 3). However, there were insignificant differ&shy;ences between nurses&rsquo; response with medication information and dietary infor&shy;mation (p value&gt;0.05).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The findings of this study revealed that nurses in CCU/CRW have identified the four most important areas of learning needs of post myocardial infarction pa&shy;tients as: symptom management, medi&shy;cation information, lifestyle factor, and anatomy and physiology. However, nurses in Medical Ward 1 and 2 ranked the four most important areas of learning needs of post myocardial infarction pa&shy;tients as: medication information, symp&shy;tom management, psychological factors and finally dietary factors. In comparison, Karlik et al. (1990) found that patients identified life style factors, medication, and activity information as the most im&shy;portant learning needs. Their study also concluded that registered nurses ranked medication information, psychological factors, and physical activity information as the most important leaning needs (Kardik et al. 1990). The findings from these studies consistently indicated that post myocardial infarction patient chose lifestyle factors as one of the top three important learning needs while registered nurses did not (Chan et al. 1990, Kardik et al. 1990). According to Casey et al. (1984), 12 physicians, 33 nurses and 30 patients completed questionnaires in or&shy;der to elicit similarities and differences in their perception of educational needs of patients&rsquo; post MI. This study reported that general agreement existed between the three groups, since they all felt that dealing with emotions, medications, and risk factors were the most important ar&shy;eas of information needs. With regard to post MI patients in the hospital, Moynihan (1984) reported that the four most important areas of information in ascending importance were risk factors, activities, medications and diet.</p>\r\n<p>\r\n	In this study, symptom management was ranked top priority among nurses in CCU/CRW but in Medical Ward 1 and 2, it was ranked as second. One possible reason why symptom management was ranked higher by nurses may be pre&shy;sumed as early detection, therefore pa&shy;tient would be able to respond better with treatment and to reduce anxiety and aid recovery. The survival rate will be higher and the survivor would have better qual&shy;ity of life. However, judging patient infor&shy;mation needs vary depending on the nurses rendering the nursing care and the influencing factors present such as the illness, severity, urgency, non-urgent and whether non-life threatening or life threatening (Hallstrom, et al. 2001). Ac&shy;cording to Timmins et al. (2003), it has been observed that nurse and patients tend to evaluate patients information needs differently. Some studies have shown nurses tend to underestimate pa&shy;tient information needs from physical and emotional perspective (Burney et al. 2002, Farrel 1991). The research findings of Hanssen et al. (2004) and Lauiri et al. (1997) revealed that nurses underesti&shy;mate patient needs which included infor&shy;mation on illness and hospital process, apart from environmental needs which is congruent with this study.</p>\r\n<p>\r\n	Amongst nurses who were surveyed in this study, 60% had three to five years of experience. The length of services of nurses has great influence on their abili&shy;ties in decision making. A nurse who has less number of years of experience, may not be able to identify adequate informa&shy;tion most ideal to be delivered to the post myocardial infarction patient. According to Benner (2000), a novice nurse at her initial stage of development, has no ex&shy;perience in real life situation. As an ad&shy;vanced beginner, a nurse gradually has experience in real life situations and can recognize different elements. They tend to focus on tasks and routines rather than on patient care. A competent nurse has acquired adequate knowledge and ex&shy;perience and has the ability to deal with elements in a situation by organizing them hierarchically; draws on past ex&shy;perience and is able to increase her in&shy;volvement with patients. A proficient nurse is able to operate more intuitively; can discriminate in difficult situations. An expert nurse possess the intuitive grasp of a situation based on past experience; knows what to do from individual patients perspective as well as from patients&rsquo; condition. Several studies also reported similar findings related to patient educa&shy;tion and appropriateness information to be disseminated (Timmins 2005, Hallstrom &amp; Elander 2001; Adam, 2002).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	This study concluded that the information needs are not always perceived in unison by nursing personnel as the CCU and CRW nurses ranked symptoms man&shy;agement as the cardinal factor whilst Medical Ward 1 and 2 nurses give para&shy;mount superiority to medication. In order to improve this situation for post MI pa&shy;tient, information need has to be tailored, individualized and prioritized based upon their needs. This will enable post myo&shy;cardial infarction patients to find a meaningful way to adopt a healthier life&shy;style and understand the importance of risk factors reduction.</p>\r\n', 1),
(420, 417, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Pregnancy anaemia is defined by hae&shy;moglobin (Hb) of less than 110g/l in the first or third trimester or less than 105g/l in the second trimester. Iron deficiency with or without anaemia has many ad&shy;verse effects on the nervous system, in&shy;tellectual capacity, physical performance, immune response and pregnancy out&shy;come(Fairbanks &amp; Beutler1995, Baynes1994). The mother may be overwhelmed by easy fatigability, dyspnoea, headache, reduced exercise tolerance and weak&shy;ness. On the other hand, the fetus may encounter an increased risk of low birth weight and preterm delivery.</p>\r\n<p>\r\n	Up to 30% of women in the post-partum period are affected by anaemia of under 10g/dL and about 10% by severe anae&shy;mia of less than 8%. By far the main cause for this is the deficiency of iron supplied during pregnancy and blood loss during and after birth(Lebrecht et al. 1995).Under normal circumstances, oral iron is the treatment of choice since it is simple, effective, safe and cheap. How&shy;ever, oral iron is better absorbed in an empty stomach but patient may experi&shy;ence side effects such as nausea, vom&shy;iting, gastritis, diarrhea and constipation. These side effects can be minimized by co-administration with food, although this may substantially decrease absorption, particularly of ferrous preparation(Perewusnyk et al. 2002). Even a patient who responds well to oral iron therapy would require a long time, usually months to reach the target haemoglobin level. This means that they have to suffer from iron deficiency for extended periodsun&shy;necessarily(Al-Momen et al. 1996).</p>\r\n<p>\r\n	A pregnant woman without anaemia may require at least 1000mg of elemental iron to be delivered to the haemopoietic organs while the anaemic patient may need more than 2600mg. This require&shy;ment cannot be met by the oral route in the majority of patients because of limited absorption, bio-availability and compli&shy;ance(Bynumet al. 1977, Lee &amp; Feldman1993, Kahrilas &amp; Hogan1993).</p>\r\n<p>\r\n	Parenteral iron therapy with intravenous iron-sucrose complex (Venofer) was first introduced to UKMMC in 2002.&nbsp; It has been used to correct anaemia in preg&shy;nant women in whom the elemental iron requirement cannot be met by oral route. Previous studies have shown that intra&shy;venous Venofer is safe and effective in pregnant and post-partum women with iron deficiency anaemia(Breymann et al. 2000, Al-Momen et al. 1996).</p>\r\n<p>\r\n	<strong>OBJECTIVE</strong></p>\r\n<p>\r\n	The aim of this study was to assess if administration of intravenous iron su&shy;crose complex (Venofer) was a sensible option in treating iron deficiency anaemia in pregnancy and puerperium using day&shy;care clinic in an urban hospital. A favor&shy;able outcome would imply its safe usage as an outpatient for treating anaemia in pregnancy.&nbsp;</p>\r\n<p>\r\n	<strong>METHODOLOGY</strong></p>\r\n<p>\r\n	This was a cross-sectional observational study, recruiting all patients with iron de&shy;ficiency anaemia who were referred to the Obstetrics Day Care Unit, UKMMC for intravenous Venofer. The study was completed in 18 months from March 2003 to August 2004. Baseline haemo&shy;globin (Hb) level was determined and reassessed in 14 daysafter treatment. Patients&rsquo; demography, number of visits to the Day Care Unit, total dosage, incre&shy;ment of haemoglobin level, adverse ef&shy;fect and their compliance to complete the full therapy were recorded. Patients with other causes of anaemia such as thalas&shy;semia trait, haemolytic anaemia, liver or renal disease were excluded.</p>\r\n<p>\r\n	<em>Calculation of Venofer dosage</em></p>\r\n<table border="1" cellpadding="1" cellspacing="1" height="38" width="533">\r\n	<tbody>\r\n		<tr>\r\n			<td>\r\n				Total iron deficit (mg) = [Body weight (kg) x (target Hb&ndash;actual Hb)g/l x 0.24*]+ depot iron (mg)</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p style="margin-left:22.5pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:22.5pt;">\r\n	For body weight of above 35 kg, the tar&shy;get Hb is 120 g/l and depot iron is 500mg.&nbsp;&nbsp;</p>\r\n<p style="margin-left:22.5pt;">\r\n	*Factor = 0.0034 x 0.07 x 1000</p>\r\n<p>\r\n	(iron content of Hb ~0.34%; Blood vol&shy;ume ~7% of body weight; factor 1000 is conversion from g to mg)</p>\r\n<p style="margin-left:22.5pt;">\r\n	<em>Administration of Venofer</em></p>\r\n<p>\r\n	The first 25mg of iron (i.e. 25mls of solu&shy;tion) was infused as a test dose over a period of 15 minutes. If there were no adverse events the remaining portion of the infusion was given. Infusion of 200mg iron sucrose in 200ml normal saline over 1 hour 30 minutes was administered at an interval of 1 to 3 days to complete the total dose required.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 120 patients were recruited. There were 70% Malay, 16% Chinese, 10% Indian and 4% from other ethnic background. About half the patients (48%) were working mothers, 45% were housewives and 7% were students.</p>\r\n<p>\r\n	The mean gestational age of those who were referred for anaemia in pregnancy was at 34.5 weeks. The pre-treatment Hb level was 8.5<u>+</u>0.85g/dl (7.2-8.9g/dl). In the post-partum group, the pre-treatment Hb was 7.6<u>+</u>0.80g/dl (7.2-8.8g/dl). Major&shy;ity of these patients (92%) had a history of either non-compliance or intolerable to oral iron supplement, hence were offered intravenous Venofer. The other 8% re&shy;quired rapid restoration due to recent blood lost from post-partum haemor&shy;rhage.</p>\r\n<p>\r\n	Of those who completed treatment, the mean increment of Hb within 14 days was 3.52g/dl (2.9-4.6g/dl). Each patient received an average of seven ampoulesi.e. 700mg of Venofer.&nbsp;</p>\r\n<p>\r\n	Majority of the patients tolerated the treatment well with no discomfort or any significant complaint (Table 1). One pa&shy;tient developed skin rashes while on in&shy;fusion. She responded to intravenous Chlorpheniramine Maleate (Piriton) and the Venofer was completed at a slower infusion rate. Another patient had low-grade pyrexia of 38<sup>o</sup>C after receiving her second dose and the fever settled with Paracetamol. Seven patients complained of having a mild metallic taste and 15 patients had nausea. There were no other undesirable effects such as head&shy;ache, dizziness, palpitations, dyspnoea, vomiting, abdominal pain, diarrhoea, muscle cramps, myalgia, shivering, chest pain, injection site pain, anaphylactic re&shy;action or hospitalization for further treat&shy;ment.</p>\r\n<p>\r\n	Of the 120 patients who were recruited for the study, 10 patients (8.3%) did not complete their therapy - eight of them were antenatal mothers who delivered before completion of treatment; another two post-partum patients defaulted their follow-up.</p>\r\n<p>\r\n	Venofer was approved as a formulary item and patients received free supply be it as an in or outpatient. However, the ward charge for an in-patient in UKMMC was RM45 per day. As most patients re&shy;quired at least a three-day infusion, it was estimated that each patient might save up to RM135. Our estimation was that during the study period on the 120 outpatients, a total of RM 16,200 hospi&shy;talization fee was avoided.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Anaemia is a common deficit in preg&shy;nancy. Though traditionally it is treated by oral haematinics, some may require parenteral iron due to poor compliance to oral supplement, poor absorption or in&shy;tolerable gastro-intestinal side effects. Prior to year 2002, intra-muscular prepa&shy;ration iron dextran (Imferon) was the available parenteral preparation in UKMMC&nbsp; However, iron dextran is to be discouraged because of its adverse ef&shy;fects including pain at injection site, ir&shy;regular absorption, staining of buttock and rarely, malignancy(Martini et al. 1994, Roberson1973). &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Parenteral iron using iron-sucrose com&shy;plex (Venofer)has largely replaced iron dextran. Numerous reports from Euro&shy;pean countries have proven its effective&shy;ness and patient tolerability (Al-Momen et al. 1996, Perewusnyk et al. 2002). The results of ourobservational study have shown that iron-sucrose complex is safe, is well tolerated and effective in treating anaemic pregnant and postpartum pa&shy;tients in a day care setting. There were 120 patients in this study, twenty-four sustained minor adverse effects such asskin rash,low-grade pyrexia,metallic tasteand nausea.There was no serious side effects or anaphylactic reactions reported.</p>\r\n<p>\r\n	Iron-sucrose complex releases ironto the iron-binding proteins with a half-life of about 6 hours. This relatively short half-lifegrants iteffectiveand carries minimal risk of allergic reaction(Danielson1998, Kraftet al. 2000, Rohlinget al. 2000). Skinrash and fever can be prevented by giving a slow infusion over one to four hoursordividing the total dose into smaller portions(100-200mg/day).Theside effects are mostly self-limited and resolve with symptomatic treatment.</p>\r\n<p>\r\n	Unlike other parenteral iron prepara&shy;tions, iron-sucrose complex istaken up mainly by the reticuloendothelialsystem and notby the parenchymal cells of the liver, kidney, adrenal gland or other or&shy;gans.Therefore, organic toxicity such as pancreatic, myorcardial or hepatic he&shy;mosiderosis is less likely even with iron-sucrose complex over-load (Macdougall et al. 1989).</p>\r\n<p>\r\n	Majority of the patients (92.8%) in this study completed the treatment course. The local urban population around UKMMC has proven themselves to be highly compliant to outpatient treatment, regardless of their background as housewives, working mothers or stu&shy;dents. Outpatient treatment allows less interruption to family or working life, is more convenient and cost saving. The avoidance of hospitalization could itself encourage patients&rsquo; compliance.</p>\r\n<p>\r\n	Although iron-sucrose complex is ex&shy;pensive and the treatment requires intra&shy;venous infusion and is time consumingas compared to oral iron supplement, it is highly tolerated and rapidly effective.This study showed thatthe mean increment of Hb within 14 days was 3.52g/dl (2.9-4.6g/dl). On the contrary, oral iron takes a longer time to achieve the target hae&shy;moglobin level and the increment rate is significantly slower (Al-Momen et al. 1996). It is very important to correct an anaemic pregnant patient before delivery, as the maternal iron status will influence the newborn iron status as well. Infants of anaemic mothers are more likely to be&shy;come anaemic at 12 months of age, even when possible confounders such as socio-economic status, feeding practices and morbidity were taken into account(Colomer et al.1990). In conclusion, out&shy;patient treatment of anaemia in preg&shy;nancy and post-partum period using Venofer is safe, feasible, with high pa&shy;tient compliance and saves cost from hospitalization fees.</p>\r\n', 2),
(421, 418, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	<em>Staphyloccocus aureus</em>is the common&shy;est cause of surgical site infection (SSI) and Methicillin Resistant <em>Staphylococcus aureus</em>(MRSA) is an important cause of SSI amongst vascular surgical patients<strong>. </strong>It is associated with poor outcome in the form of limb loss and mortality. Graft morbidity and mortality rates were re&shy;corded at 25% and 11% respectively (Surveillance of Surgical Site Infection in English Hospitals 1997-1999, 2000; Naylor et al. 2001).</p>\r\n<p>\r\n	Numerous factors have been attributed to the emergence of MRSA. These are advanced age, cigarette smoking and immunocompromised state such as dia&shy;betes melitus. Other pertinent surgical issues that may contribute to the overall outcome in MRSA patients include, the higher prevalence of peripheral vascular disease in the lower limbs, types of graft infected, long duration of surgery and the judicious use of pre-operative vancomy&shy;cin prophylaxis.</p>\r\n<p>\r\n	An increasing trend of MRSA carrier and infection had been reported in the hospitals of Europe and the United States especially in emergency cases (Muralidhar et al. 2006). We postulate a similar increasing trend of MRSA carrier and infection in Malaysia as the numbers of emergency vascular cases are equally as much.</p>\r\n<p>\r\n	The aim of this study was to review the prevalence and outcome of MRSA infec&shy;tion in the vascular unit in Hospital Kuala Lumpur (HKL). In addition, we also re&shy;viewed the associated risk factors of MRSA infection and choices of antibiotics used to treat it.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	We conducted a review of post-surgical vascular cases complicated by MRSA infection in the Vascular Surgical Unit in Hospital Kuala Lumpur (HKL) from 1st January 2005 to 31st January 2007. MRSA infections were clinically diag&shy;nosed and confirmed by microbiological study from local wounds, nasal swabs, infected grafts and blood cultures. The strains with oxacillin (&ge;1&mu;g) zone diame&shy;ter &lt;13mm diameter and/or the strains with oxacillin minimum inhibitory concen&shy;tration (MIC) value &ge;4&mu;g/ml were de&shy;scribed as Methicillin-Resistant. The risk factors studied were patients&rsquo; demogra&shy;phy, co-morbidities and cigarette smok&shy;ing habits. Previous hospitalization, sur&shy;gery, and the use of broad-spectrum an&shy;tibiotics were taken as within one year prior to the primary surgery performed. Other features examined were the type and the anatomical site of specimen taken, patients&rsquo; location of where the samples were taken and the antibiotics sensitivity results. Operative factors were the urgency of the surgery, the types of operation and the types of grafts used. We classified post-operative vascular graft infection by using the Szilagyi&rsquo;s classification which categorizes the grades of infection according to the ana&shy;tomical layer.</p>\r\n<p>\r\n	The outcome measured were length of hospital stay, incidence of sepsis, am&shy;putation rate and mortality. Prolonged hospitalization was defined as hospital stay of 24 days or more from the first day of MRSA positive swab until patients were discharged or succumbed. The complications and outcomes were taken as those occurred within the same hos&shy;pital stay. Complications such as wound breakdown, graft dehiscence, graft thrombosis and pseudo-aneurysm were also reviewed.</p>\r\n<p>\r\n	Statistical analysis was performed us&shy;ing the SPSS version 12.0 and tests such as Chi-square and Fisher&rsquo;s Exact Test whenever appropriate. The P value of less than 0.05 was considered signifi&shy;cant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	In total, there were 999 vascular surger&shy;ies in HKL within the study period. Of these, 24 patients (2.4%) were compli&shy;cated by MRSA infection. The male to female ratio was 2:1 with the median age of 60 years old.</p>\r\n<p>\r\n	Diabetes melitus was found in 11 pa&shy;tients infected with MRSA, while previous history of peripheral vascular disease was seen in 8 patients. Twelve patients (50.0%) were chronic cigarette smokers at the time of hospitalization.</p>\r\n<p>\r\n	Multiple hospitalizations with previous surgery were characteristic of MRSA in&shy;fection in 10 of these patients (41.7%). Fifteen patients (62.5%) had previous broad-spectrum antibiotic usage and 15 patients (62.5%) had history of previous hospitalization (Table 1).</p>\r\n<p>\r\n	The sites of positive cultures were from various sources. However, there was no positive culture detected in any of the vascular grafts used. In addition, the lower limbs yielded the highest rate of<br clear="all" />\r\n	positive MRSA culture.</p>\r\n<p>\r\n	As for the location of the patients, ma&shy;jority of the positive MRSA swabs were obtained from the general wards (70.8%), followed by the isolation rooms (16.7%) and ICUs (12.5%).</p>\r\n<p>\r\n	There was a higher prevalence of in&shy;fection in emergency cases with forty-six percent of the infected cases were done as emergency surgery compared to 37.5% elective and 16.7% urgent cases. The breakdown of each type of vascular surgery performed is shown in Table 2.</p>\r\n<p>\r\n	In two-thirds of the MRSA infected cases, vascular grafts were used. The breakdown of the types of grafts is shown in Table 2. Of these, higher infection rate was seen in patients using prosthetic graft (5 Dacron, 4 PTFE) as compared to those with venous graft (n=5). Two other cases used composite graft. However, the presence of vascular graft did not predict poor outcome of MRSA infection (p=0.679).</p>\r\n<p>\r\n	Of the 24 MRSA infected patients, 14 patients (58.3%) developed wound infec&shy;tion (5 superficial, 6 deep, 3 exposing grafts). In addition, one case had super&shy;imposed graft dehiscence and a pseudo-aneurysm respectively. Prolonged hos&shy;pitalization was recorded in one-third of the patients (n=8). These tend to develop complications (p=0.006) and have poorer outcome (p=0.033) such as limb loss and mortality.</p>\r\n<p>\r\n	As for the treatment of MRSA infection, five patients were treated with Vancomy&shy;cin alone, and 10 patients were treated with both Vancomycin and surgery such as wound debridement, graft removal, sartorius flap usage or amputation. The remaining patients (n=7) were neither treated with Vancomycin nor surgical treatment but were given other types of antibiotics such as Teicoplanin or Ri&shy;fampin according to culture and sensitiv&shy;ity result; Mupirocin for nasal carriage.</p>\r\n<p>\r\n	Poor outcome was recorded in 13 out of 24 patients (54.2%). Of these, 4 pa&shy;tients (16.7%) died of MRSA infection while the other 9 patients developed mul&shy;tiple complications such as graft removal (4 out of 9 patients, 44%), and sepsis (5 out of 9 patients, 56%). Out of these 9 patients, 6 required lower limb amputa&shy;tion.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Our results revealed the debilitating ef&shy;fects of MRSA infection in vascular pa&shy;tients. The prevalence of MRSA infection in vascular patients was low at 2.4% over a period of two years. This low rate could be attributed to the preventive measures, which included prophylactic antibiotic used in selected high risk patients, bar&shy;rier nursing and universal precaution.</p>\r\n<p>\r\n	However, a high percentage of chronic cigarette smokers and patients with dia&shy;betes melitus were obvious in this subset of patients. Smokers and diabetics are found to be predictors of harbouring MRSA infection (Scott et al. 2005)<strong>. </strong>This trend seemed to be similar in another report by Thompson (2006).</p>\r\n<p>\r\n	With regards to the anatomical sites of positive culture, lower limb swabs usually reflects local wound infection of an ischemic wound and is often described in patients with prolonged hospitalization. The significance of a positive culture is not great to warrant local wound treat&shy;ment with debridement and dressing. This is contrary to abdominal or groin wound infections that are actually surgi&shy;cal site infections.</p>\r\n<p>\r\n	The most prominent finding in this study is the poor outcome in MRSA infected patients. The mortality rate was 16.7% and these patients mostly died of septi&shy;caemia eventually, as a complication of MRSA infection. On the other hand, seri&shy;ous morbidity was encountered in 37.5 % of the infected patients in the form of limb amputation and graft removal. In other words, more than half of these patients were at risk of either mortality or serious complications. Gassel et al (2002) re&shy;ported 17.0% mortality, which was similar to our study.</p>\r\n<p>\r\n	The mortality will depend on the source and site of infection, with aortic MRSA infection recorded nearly 100.0% mortal&shy;ity. Murphy et al concluded that graft in&shy;fection posed a catastrophic complication with mortality reaching up to 56.0% in aorto-illiac reconstruction (Murphy et al. 2001). This brings us to the importance of preventive measures that must be taken seriously to avoid such morbidities caused by MRSA infection. Serious com&shy;plications with 25.0% resulting in major amputation and 11.0% mortality rate (Gemmel et al. 2006).</p>\r\n<p>\r\n	The rationale behind various treatment modalities can be grouped into patient or operative factors. Patients with co-mor&shy;bid, for instance, uncontrolled diabetes melitus and peripheral vascular disease would progress to poorer or delayed wound healing. Timing of surgery ac&shy;counts for higher rates of MRSA infection in emergency cases whereby inadequate antibiotic prophylaxis, and lack of time for careful pre-operative assessment and planning, contributed in one way or an&shy;other. Thus, treatment option depends on patient-to-patient basis and good surgical skills. Generally, daily dressing with wider antibiotic coverage is the initial therapy before aggressive treatment with graft removal or amputation intervention. However, the reverse may be life saving. This happens in gangrenous limbs, es&shy;pecially wet gangrene whereby amputa&shy;tion is the treatment of choice. Infected graft unresponsive to antibiotics will eventually need a graft revision. How&shy;ever, this depends on the site of the initial graft as reoperation at the abdominal aortic aneurysm (AAA) or the inguinal region can be devastating secondary to its highly infected-prone area and pa&shy;tient&rsquo;s age, co morbidities and risks of deep vein thrombosis or pulmonary em&shy;bolism. A re-operation of AAA can achieve near 100% mortality and should be borne in mind.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In conclusion, our small study has proven that MRSA infection brings about unfa&shy;vorable outcome to vascular patients. Preventive measures need to be up&shy;graded to battle the increasing trend of MRSA infection. In our attempt to seek for the factors affecting MRSA infection, we failed to identify specific predictors. This is largely attributed to the small sample size that we reviewed. Defined predictors for MRSA infection is yet to be established but several co-morbidities such as diabetes melitus, chronic smok&shy;ing, multiple previous hospitalization and previous broad-spectrum antibiotics used tend to be more prevalent in MRSA in&shy;fection.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(422, 419, '<div>\r\n	<p>\r\n		<strong>PENDAHULUAN </strong></p>\r\n	<p>\r\n		Girus Heschl (HG) atau BA41 adalah ka&shy;wasan pemprosesan maklumat auditori primer di samping BA22 dan 42. Pengak&shy;tifan bilateral telah dapat dikesan pada HG dalam subjek berpendengaran nor&shy;mal (Penhune et al. 1996, Rademacher et al. 2001). Selain itu, subjek normal juga menunjukkan pengaktifan bilateral pada kawasan medial dan lateral HG, bahagian anterolateral planum temporal (PT) dan bahagian posterior girus tempo&shy;ral superior (STG) (Warren et al. 2005). Kajian yang menggunakan imbasan to&shy;mografi panca&shy;ran positron (PET) (Karbe et al. 1995) juga mendapati bahawa ter&shy;dapat pengaktifan bilateral pada HG, PT, STG lateral dan sulkus temporal superior (STS) akibat peningkatan aktiviti metabo&shy;lism dalam otak semasa subjek mende&shy;ngar bunyi stimulus. Dalam kajian lain (Brugge et al. 2003) yang menggunakan stimulus elektrik ke atas pesakit yang menghidap epilepsi, didapati HG dan STG terlibat dalam kefungsian auditori dengan kehubungan kefungsian berlaku di antara HG dan STG posterior. Kajian yang dijalankan (Hart et al. 2002) untuk melihat respons kawasan auditori primer dan bukan primer terhadap stimulus yang diberikan iaitu bunyi dengan be&shy;berapa aras keamatan untuk menentu&shy;kan kawasan auditori primer dan bukan primer, mendapati bahawa terdapat peningkatan magnitud respons hemodi&shy;namik pada HG dengan peningkatan aras keamatan stimulus. Selain itu, HG ternyata lebih dominan dalam pem&shy;prosesan bunyi de&shy;ngan pelbagai aras pendengaran, ber&shy;banding dengan dua lagi kawasan auditori bukan primer iaitu kawasan anterolateral kepada HG dan planum temporal. Kajian ke atas subjek berpendengaran normal mendapati ba&shy;hawa tahap pengaktifan pada STG meningkat dengan peningkatan kea&shy;mat&shy;an stimulus (J&auml;ncke et al. 1998, Strainer et al. 1997). Subjek berpende&shy;ngaran normal yang menerima stimulus pada 75, 85 dan 95 dB didapati menunjuk&shy;kan pengaktifan yang lebih besar pada STG dengan peningkatan keamatan stimulus hingar putih.</p>\r\n	<p>\r\n		Sungguhpun telah banyak kajian fMRI yang menggunakan stimulus hingar di&shy;jalankan ke atas korteks auditori manu&shy;sia, masih banyak lagi yang belum difa&shy;hami terutamanya dalam konteks or&shy;ganisasi ke&shy;fungsian korteks auditori iaitu pengkhususan dan persepaduan kefung&shy;sian. Sifat persepaduan kefungsian iaitu kehubungan kefungsian dan kehubungan efektif antara kawasan utama dalam kor&shy;teks auditori pula misalnya, memerlukan maklumat mengenai ketinggian dan kelu&shy;asan pengaktifan yang hanya dapat diperolehi melalui kajian pengkhususan kefungsian.</p>\r\n	<p>\r\n		Analisis data fMRI dalam kajian ini mem&shy;punyai dua objektif iaitu 1) menge&shy;nalpasti kawasan otak yang terlibat dalam pende&shy;ngaran dan 2) menentukan kesan saiznya iaitu merujuk kepada keamatan dan luas kawasan pengaktifan. Kajian ditumpukan kepada penentuan sifat pengkhususan kefungsian dalam korteks auditori subjek berpendengaran normal. Stimulasi auditori yang sama iaitu hingar putih diberikan kepada se&shy;mua subjek secara binaural se&shy;masa im&shy;basan fMRI dijalankan. Hingar putih digunakan dalam kajian ini kerana hingar jenis ini mempunyai spektrum kuasa dengan jalur lebar jangka-panjang yang dapat menguja sebilangan besar gentian saraf auditori (Gour&eacute;vitch et al. 2008). Se&shy;lain itu, perbezaan kawasan pengkhususan kefungsian pada korteks auditori bagi kumpulan subjek ini juga akan dikenalpasti. Sifat pengkhususan kefungsian tersebut ditentukan meng&shy;gunakan kaedah analisis pemetaan sta&shy;tistik berparameter (SPM). Kajian ini dianggap sebagai pra-keperluan kepada kajian lanjutan ke atas sifat ke&shy;hubungan efektif di antara kawasan kor&shy;teks auditori primer yang akan diterbitkan dalam pe&shy;nerbitan berasingan.</p>\r\n	<p>\r\n		<strong>BAHAN DAN KAEDAH</strong></p>\r\n	<p>\r\n		Kajian dilakukan ke atas 5 orang subjek lelaki berbangsa Melayu berumur 20 hingga 40 tahun. Ujian pendengaran yang dijalankan di Jabatan Audiologi dan Sains Pertuturan (JASP), Fakulti Sains Kesihatan Bersekutu (FSKB), Universiti Kebangsaan Malaysia (UKM) menge&shy;sahkan bahawa kesemua subjek ber&shy;pendengaran normal dan mempunyai keadaan telinga tengah yang baik. Ujian pendengaran tersebut dikendalikan oleh ahli audiologi dengan menggunakan nada asli dalam julat fre&shy;kuensi 250 Hz hingga 8000 Hz. Subjek normal perlu mempunyai aras pendengar&shy;an (HL) tidak lebih daripada 30 dB (Langers et al. 2005). Kajian yang di&shy;jalankan ini telah mendapat kelulusan Jawatankuasa Pen&shy;yelidikan dan Etika Penyelidikan Peru&shy;batan (JKPEPP), UKM (No. rujukan: FF-205-2006).</p>\r\n	<p>\r\n		<em>Imbasan fMRI</em></p>\r\n	<p>\r\n		Imbasan fMRI dijalankan dengan meng&shy;gunakan mesin pengimbas MRI 1.5 tesla berjenama Siemens Magnetom Avanto di Jabatan Radiologi, Pusat Perubatan Uni&shy;versiti Ke&shy;bangsaan Malaysia (PPUKM). Subjek di&shy;baringkan secara supin di atas katil MRI dan dipasangkan dengan gegelung RF kepala. Subjek dipakaikan dengan fon kepala untuk menerima arahan dan bunyi hingar putih yang diperdengarkan secara binaural. Subjek diminta menumpukan perhatian dan memberi tindakbalas de&shy;ngan menekan bebuli getah mengguna&shy;kan ta&shy;ngan kanan apabila mendengar bunyi hi&shy;ngar. Hingar putih ditetapkan pada 70 dB lebih tinggi daripada aras pendengaran manusia normal. Imbasan yang dilakukan adalah pada kawasan yang merangkumi keselu&shy;ruhan korteks auditori (Ahmad Nazlim Yusoff et al. 2008, Aini Ismafairus Abd Hamid et al. 2008, Mazlyfarina Mohamad et al. 2008).</p>\r\n	<p>\r\n		<em>Paradigma ujikaji</em></p>\r\n	<p>\r\n		Paradigma fMRI senyap (Rajah 1a) digunakan untuk mengurangkan artifak pada imej akibat bunyi imbasan MRI (Yetkin et al. 2004). Paradigma jenis ini membenarkan respons hemodinamik yang terhasil daripada stimulasi yang diberikan, diukur dengan tepat tanpa di&shy;ganggu oleh respons hemodinamik bunyi imbasan pen&shy;gimejan gemaan satah (EPI) yang dihasil&shy;kan oleh mesin pengimbas MRI. Umum&shy;nya, berlaku suatu sela masa finit di antara pemberian stimulus dan pengaktifan otak. Untuk respons visual, deria dan motor, puncak respons hemodinamik berlaku dalam masa 5-10 saat. Puncak respons hemodinamik auditori pula mengambil masa yang agak lama untuk berlaku iaitu di sekitar 10.8 s (Bandettini et al. 1993). Paradigma yang digunakan dalam kajian ini telah meng&shy;ambil kira kelakuan respons hemodi&shy;namik auditori ini (Aini Ismafairus Abd Hamid et al. 2008, Mazlyfarina Mohamad et al. 2008).</p>\r\n	<p>\r\n		<em>Pemprosesan data fMRI</em></p>\r\n	<p>\r\n		Kesemua imej kefungsian (berwajaran T2*) dan struktur (berwajaran T1) disim&shy;pan dalam cakera padat untuk dianalisis (Ahmad Nazlim Yusoff et al. 2008). Analisis imej dijalankan menggunakan komputer peribadi dengan kelajuan pem&shy;prosesan yang tinggi dan ruang penyim&shy;panan data yang besar. Sifat pengkhususan kefung&shy;sian korteks audi&shy;tori ditentukan meng&shy;gunakan pakej peri&shy;sian MATLAB 7.4 versi 2007a (Mathworks Inc., Natick, MA, USA) dan Pemetaan Statistik Berparameter (Functional Imaging Laboratory, Wellcome Department of Imaging Neuroscience, In&shy;stitute of Neurology, Universiti College of London).</p>\r\n	<p>\r\n		Daripada imej statistik yang dijana oleh SPM dan WFU PickAtlas (Bandettini et al. 1993), beberapa koordinat MNI yang menunjukkan keamatan maksimum (<em>t</em>&gt;3.5) di dalam setiap BA dikenalpasti. Koor&shy;dinat MNI tersebut kemudiannya didaftar&shy;kan di dalam perisian MarsBar (Brett et al. 2002) dan suatu sfera berje&shy;jari 8 mm yang ber&shy;pusat di setiap koordi&shy;nat MNI berkea&shy;matan maksimum terse&shy;but kemudiannya dibina. PPI bagi setiap sfera kemudiannya ditentu&shy;kan dan purata bagi PPI setiap BA di hemisfera kanan dan kiri diperolehi. PPI yang diperolehi tersebut ditakrifkan seba&shy;gai nisbah antara perubahan isyarat keadaan aktif terhadap purata isyarat bagi keseluruhan pengukuran untuk setiap BA22, 41 dan 42. Nilai yang diperolehi adalah peruba&shy;han isyarat resonans mag&shy;net relatif di dalam suatu kawasan (<em>cytoar&shy;chitecture</em>) yang berpunca daripada re&shy;spons hemodinamik apabila subjek mendengar hingar putih (Eickhoff et al. 2005).</p>\r\n	<p>\r\n		<strong>KEPUTUSAN</strong></p>\r\n	<p>\r\n		<em>Imej statistik otak</em></p>\r\n	<p>\r\n		Rajah 2 adalah peta statistik berpara&shy;me&shy;ter (SPMs) yang menunjukkan pro&shy;jeksi kea&shy;matan maksimum (MIP) bagi imej sta&shy;tistik otak kesemua subjek yang dipapar&shy;kan pada koordinat MNI. Koordi&shy;nat MNI adalah ruang stereotaktik piawai yang di&shy;sediakan oleh McConnel Brain Imaging Center, Montr&eacute;al Neurological Institute, McGill Uni&shy;versity, untuk kegunaan pe&shy;metaan pen&shy;gaktifan otak &nbsp;(Ashburner &amp; Friston 2004). MIP yang ditunjukkan adalah mengikut ta&shy;tacara neurologi dalam pembacaan imej iaitu sebelah kanan imej adalah bahagian kanan otak. SPMs adalah imej statistik yang men&shy;gandungi maklumat mengenai lokaliti, ke&shy;luasan dan keamatan isyarat pengaktifan otak. SPMs dalam Rajah 2 menunjukkan imej statistik <em>t </em>yang telah dijana meng&shy;gunakan perisian SPM dan WFU PickAtlas (Maldijan et al. 2003), supaya kawasan pengaktifan yang dikehendaki hanya ditak&shy;rifkan pada kawasan anatomi Brodmann (BA) 22, 41 dan 42 (Kawasan Brodmann ditakrifkan oleh Korbinian Brodmann (Brodmann 1909) dan mem&shy;punyai nom&shy;bor 1-52 (Rajah 1b). Kawa&shy;san ini diperolehi berdasarkan organisasi kor&shy;teks yang diperhatikan apabila tisu otak diwarnakan untuk mengenalpasti sel saraf tertentu &ndash; <em>cytoarchitecture</em>). Ini dila&shy;kukan mengguna&shy;kan pembetulan voksel kecil (SVC) de&shy;ngan menapis keluar ka&shy;wasan yang tidak terma&shy;suk dalam kor&shy;teks auditori primer meman&shy;dangkan ob&shy;jektif kajian ini hanya tertumpu kepada kawasan tersebut sahaja. Kelom&shy;pok vok&shy;sel (<em>volume element</em>) ber&shy;warna hi&shy;tam-kelabu menunjukkan dar&shy;jah ketinggian isyarat resonans magnet atau pengaktif&shy;an otak dengan semakin tinggi magnitud pengaktifan, semakin tinggi nilai <em>t</em> bagi vok&shy;sel tersebut dan semakin hitam suatu vok&shy;sel. Simbol (<strong>&lt;</strong>) berwarna merah menanda&shy;kan voksel yang mempu&shy;nyai keamatan maksimum. Dapat diper&shy;hatikan bahawa pengaktifan otak kesemua subjek adalah secara bilateral tak simetri dengan kese&shy;mua titik maksimum terletak pada hemi&shy;sfera kanan otak.</p>\r\n	<p>\r\n		<em>Peratus perubahan isyarat</em></p>\r\n	<p>\r\n		Jadual 1 menunjukkan purata peratus pe&shy;rubahan isyarat (PPI) yang berlaku pada BA22, 41 dan 42 di kedua-dua hemisfera kanan dan kiri otak. PPI bagi setiap BA (22, 41 dan 42) di kedua-dua hemisfera ditentukan untuk kesemua subjek. Kepu&shy;tusan yang diperolehi adalah BA22, 41 dan 42 di hemisfera kanan menunjukkan nilai purata PPI yang lebih tinggi berbanding dengan BA22, 41 dan 42 di hemisfera kiri. Bi&shy;langan data PPI (<em>N</em>) untuk setiap BAyang dijana oleh SPM. Ini berlaku dise&shy;bab&shy;kan oleh ciri respons auditori yang secara semulajadinya kecil jika diban&shy;dingkan de&shy;ngan respons lain dalam otak misalnya motor, ingatan dan penglihatan. Analisis kenormalan mendapati kesemua data bagi setiap BA yang dikaji tidak ter&shy;tabur secara normal. Oleh itu, Ujian Mann-Whitney U digunakan dalam me&shy;nentukan perbezaan di antara purata nilai PPI BA kanan dan kiri. Keputusan bagi median dan julat in&shy;terkuartil diberi&shy;kan juga dalam Jadual 1.</p>\r\n	<p>\r\n		Rajah 3 menunjukkan graf korelasi li&shy;near di antara PPI bagi kelompok peng&shy;aktifan dan PPI bagi voksel dengan kea&shy;matan maksimum pada kelompok peng&shy;aktifan tersebut. Data yang dianalisis diperolehi daripada kesemua subjek dan hanya ter&shy;had kepada kelompok pengak&shy;tifan pada kawasan BA22, 41 dan 42 sa&shy;haja. Analisis dilakukan untuk voksel dengan nilai <em>t</em>&gt;3.5. Keputusan ujian sta&shy;tistik Pearson menun&shy;jukkan wujud kore&shy;lasi linear yang sangat baik (<em>r</em><sub>pearson</sub>=0.817) dan bererti (<em>p</em>&lt;0.001) di antara dua pembolehubah terse&shy;but. Ini adalah wajar kerana kesemua vok&shy;sel indi&shy;vidu di dalam kesemua kelompok pengak&shy;tifan menunjukkan PPI yang bernilai positif terhadap stimulus yang dikenakan. Kore&shy;lasi adalah baik untuk nilai PPI rendah (&lt;1.50). Walau bagai&shy;manapun, taburan data mula mencapah untuk nilai kedua-dua pemboleh ubah yang lebih besar daripada 1.50.</p>\r\n	<p>\r\n		<em>Pengkhususan kefungsian</em></p>\r\n	<p>\r\n		Jadual 2 menyenaraikan koordinat titik maksimum pada hemisfera kanan dan kiri dengan jumlah voksel yang meng&shy;alami pengaktifan (voksel aktif) di kedua-dua hemisfera tersebut untuk setiap subjek. Data diperolehi melalui analisis SPM ke atas Rajah 2 dengan hanya mengambil kira BA22, BA41 dan BA42 sahaja. Dari&shy;pada keputusan tersebut di&shy;dapati jumlah voksel aktif di kedua-dua hemisfera kanan dan kiri adalah ber&shy;lainan dan nilainya tidak sama untuk setiap subjek. Empat daripada lima orang subjek menunjukkan bilangan vok&shy;sel aktif yang lebih tinggi pada lobus temporal kanan. Subjek 4 menunjukkan bilangan voksel aktif yang lebih tinggi pada lobus temporal kiri, iaitu sebanyak 305&plusmn;1 vok&shy;sel, berbanding dengan 278&plusmn;1 voksel pada lobus temporal kanan. Purata bi&shy;lang&shy;an voksel aktif kesemua subjek un&shy;tuk lobus temporal kanan dan kiri adalah masing- masing 205&plusmn;40 dan 148&plusmn;42. Keputusan ujian statistik Mann Whitney U sekali lagi menunjukkan perbezaan purata bilangan voksel aktif yang tidak bererti (<em>p</em>&gt;0.05, z= -1.149) di antara lobus temporal kanan dan kiri dengan Median&plusmn;IQR adalah 191&plusmn;154 (95% CI; 102.91:307.99) untuk sebe&shy;lah kanan dan 139&plusmn;155 (95% CI;30.31 : 266.09) untuk sebelah kiri dengan CI=sela keyakinan. Walau bagaimanapun, jika magnitud pengaktifan otak dipengaruhi oleh kedua-dua keting&shy;gian isyarat dan ke&shy;luasan ka&shy;wasan pen&shy;gaktifan, bilangan voksel aktif yang lebih tinggi pada hemi&shy;sfera kanan yang diperolehi adalah tekal dengan keputusan untuk PPI sebelum ini. Jadual 3 menyena&shy;raikan data sta&shy;tistik, bi&shy;langan voksel aktif, koordinat MNI dan ka&shy;wasan Brodmann yang mengalami peng&shy;aktifan dengan lebih terperinci untuk ke&shy;semua subjek. Kawasan pengaktifan tersebut diperolehi dengan melakukan analisis teori medan rawak Gaussianpada aras keertian (<em>&alpha;</em>) 0.05 de&shy;ngan nilai <em>p </em>diper&shy;betulkan untuk keselu&shy;ruhan imej pada aras keertian (<em>&alpha;</em>) 0.05 dengan nilai <em>p </em>diperbetul&shy;kan untuk keselu&shy;ruhan imej statistik otak (Brett et al. 2004) meng&shy;gunakan pem&shy;betulan <em>family wise er&shy;ror</em> (FWE). Pem&shy;betulan FWE pada aras keertian 0.05, atau <em>&alpha;</em><sub>FWE</sub>=0.05, bermak&shy;sud kebarangkalian untuk mendapatkan peng&shy;aktifan yang palsu adalah 1 dalam 20 SPMs yang diha&shy;silkan. Hanya kelom&shy;pok yang mempunyai lebih daripada 20 voksel aktif pada kawa&shy;san yang diminati sahaja yang dianggap menunjukkan pengaktifan sebenar. Nilai <em>p </em>yang diper&shy;betulkan (<em>p</em><sub>FWE</sub>) dalam jadual tersebut ditakrifkan merujuk kepada infe&shy;rens aras set (jumlah kelompok pengaktif&shy;an yang bererti), inferens aras kelompok (jumlah voksel aktif secara ber&shy;erti dalam suatu kelompok) dan inferens aras voksel (vok&shy;sel individu yang meng&shy;alami pengak&shy;tifan secara bererti) (Friston et al. 1996, Friston 2004). Ketiga-tiga aras inferens ini penting dalam menentusahkan ba&shy;hawa suatu set kelompok voksel atau suatu kelompok voksel atau voksel indi&shy;vidu tersebut adalah bererti pada suatu aras keertian. Berdasarkan kepada data statistik dalam Jadual 3, dapat disimpul&shy;kan bahawa pengaktifan otak pada Rajah 2 adalah ber&shy;erti untuk ketiga-tiga aras inferens pada <em>&alpha;</em><sub>FWE</sub>=0.05.</p>\r\n	<div>\r\n		<p>\r\n			<em>Analisis FFX dan RFX</em></p>\r\n		<p>\r\n			Hasil analisis individu jelas menunjukkan kewujudan variabiliti bukan sahaja antara imbasan ke imbasan bahkan antara subjek ke subjek. Akan tetapi, amat mustahak bagi suatu kajian pengimejan sains neuro untuk membuat inferens ke atas kumpulan subjek yang dikaji (pe&shy;ringkat pertama) atau mungkin ke atas populasi daripada mana subjek dipilih (peringkat kedua). Untuk itu, satu bentuk pengaktifan otak purata diper&shy;lukan (Friston 2004). Inferens peringkat per&shy;tama dan kedua ini boleh dilakukan menggunakan analisis kesan malar (FFX) dan analisis kesan rawak (RFX). Rajah 4a dan 4b menunjukkan pengak&shy;tifan otak pu&shy;rata yang terhasil masing-masing daripada FFX dan RFX ke atas semua subjek.</p>\r\n		<p>\r\n			Jadual 4a menyenaraikan kelompok vok&shy;sel aktif, jumlah voksel aktif, nilai sta&shy;tistik, koordinat MNI <em>(x, y, z) </em>dan kawa&shy;san Brod&shy;mann (BA) yang ditakrifkan bagi setiap kelompok pengaktifan yang diperolehi daripada FFX. Seperti dalam analisis indi&shy;vidu, SPMs FFX yang ditun&shy;jukkan hanya mengambil kira pengaktifan pada kawasan BA22, 41 dan 42 pada <em>&alpha;</em><sub>FWE</sub>=0.05 dan de&shy;ngan hanya memper&shy;timbangkan kelom&shy;pok yang mempunyai 20 voksel atau lebih sa&shy;haja. Terdapat 3 kelompok voksel aktif se&shy;cara bererti. Kelompok T1 mempunyai 599&plusmn;1 voksel aktif dan berada pada lobus temporal kanan, manakala kelompok T2 dan T3 masing-masing mempunyai 462&plusmn;1 dan 23&plusmn;1 voksel aktif yang terletak pada lobus temporal kiri. Kesemua voksel dalam kelompok juga mengalami peng&shy;- aktifan se&shy;cara bererti. Lobus temporal kanan mem&shy;punyai bilangan voksel yang lebih tinggi berbanding dengan sebelah kiri. Kelompok T1 mempunyai titik de&shy;ngan keamatan maksimum pada koordi&shy;nat (56, -20, 10), iaitu pada BA41 kanan. Kelompok T2 dan T3 berlaku pada ka&shy;wasan BA42 dan BA22, masing-masing dengan titik dengan kea&shy;matan mak&shy;simum pada koordinat (-48,-14, 4) dan (-48, 12 -6).</p>\r\n		<p>\r\n			Rajah 4b dan Jadual 4b memberikan maklumat yang sama seperti di atas tetapi untuk RFX. Nilai <em>&alpha;</em> untuk SPMs ini adalah 0.01. Terdapat 9 kelompok peng&shy;aktifan. Jumlah voksel aktif adalah lebih banyak pada lobus temporal kiri, iaitu sebanyak 22 berbanding dengan di se&shy;belah kanan yang mempunyai 15 voksel. Walau bagai&shy;manapun, titik dengan kea&shy;matan mak&shy;simum terletak pada lobus temporal kanan, iaitu di BA41 pada koor&shy;dinat (58, -24, 12). Kesemua kelompok dan voksel mengalami pengaktifan se&shy;cara bererti pada aras keer&shy;tian 0.01.</p>\r\n		<p>\r\n			<em>Analisis konjunksi</em></p>\r\n		<p>\r\n			Rajah 5a dan 5b menunjukkan peta peng&shy;aktifan otak yang diperolehi dari&shy;pada anali&shy;sis konjunksi ke atas kesemua sub&shy;jek, masing-masing pada <em>&alpha;</em><sub>FWE</sub>=0.05 dan <em>&alpha;</em>=0.001. Jadual 5 pula menyenarai&shy;kan data statistik, jumlah voksel aktif yang ber&shy;erti, koordinat MNI (<em>x</em>, <em>y</em>, <em>z</em>) dan kawa&shy;san Brod&shy;mann yang berkenaan. SPMs yang terhasil pada dua aras ke&shy;yakinan ini ber&shy;beza dari segi bilangan kelompok dan vok&shy;sel aktif yang bererti. Pada <em>&alpha;</em><sub>FWE</sub>=0.05, hanya satu kelompok voksel sahaja yang mengalami pengak&shy;tifan se&shy;cara bererti (<em>p</em><sub>FWE</sub>&lt;0.05) pada aras kelompok dengan jumlah voksel aktif bererti (<em>p</em><sub>FWE</sub>&lt;0.001) pada aras vok&shy;sel sebanyak 5. Titik dengan keamatan maksimum berlaku pada koordi&shy;nat (52, -22, 6) iaitu pada BA41 kanan. Pada <em>&alpha;</em>=0.001, terdapat dua kelompok voksel aktif tetapi kedua-duanya tidak ber&shy;erti (<em>p</em>&gt;0.001) pada aras kelompok. Seba&shy;nyak 11 voksel aktif dalam kelompok per&shy;tama (T1) menunjukkan pengaktifan ber&shy;erti (<em>p</em>&lt;0.001) pada aras voksel, mana&shy;kala 6 vok&shy;sel dalam kelompok kedua menunjuk&shy;kan pengaktifan tak bererti (<em>p</em>&gt;0.001) pada aras voksel. Titik dengan keamatan mak&shy;simum untuk T1 berada pada BA41 kanan, manakala titik dengan keamatan mak&shy;simum untuk T2 berada pada BA22 kiri. Walau bagaimanapun, titik dengan kea&shy;matan maksimum untuk SPMs yang dijana pada kedua-dua aras keertian tersebut adalah sama, iaitu pada koordinat (52, -22, 6), iaitu pada BA41 kanan.</p>\r\n		<p>\r\n			<strong>PERBINCANGAN</strong></p>\r\n		<p>\r\n			Peningkatan keamatan isyarat resonans magnet yang dicerap semasa subjek mendengar bunyi hingar adalah ber&shy;punca daripada peningkatan kepekatan oksihe&shy;moglobin dalam salur darah vena (penu&shy;runan kepekatan deoksihemoglo&shy;bin) di dalam otak. Fenomena ini ber&shy;punca dari&shy;pada peningkatan pengaliran darah sere&shy;brum semasa bahagian ter&shy;tentu otak se&shy;dang dirangsang oleh stimulasi luar terse&shy;but. Kesannya, bekalan oksigen melebihi peng&shy;guna-annya lalu menyebabkan berlaku keho-mogenan medan magnet setempat di sekitar kawasan otak yang mengalami pengaktifan. Peningkatan kehomogenan medan magnet setempat akibat pening&shy;kat&shy;an kuantiti oksihemoglobin secara keselu&shy;ruhannya meningkatkan masa re&shy;laksasi T2* bagi tisu dalam rantau yang berkenaan dan sekaligus meningkatkan isyarat resonans magnet yang dicerap. Oksihe&shy;moglobin seolah-olah bertindak sebagai medium kontras dalaman yang berupaya membezakan antara bahagian otak yang diaktifkan dengan bahagian yang tidak diaktifkan apabila dirakamkan di atas imej resonans magnet T2* (Ogawa et al. 1990a, Ogawa et al. 1990b). Ini boleh berlaku disebabkan terdapat perbezaan dalam nilai keren&shy;tanan magnet bagi oksihemoglobin dan deoksihemoglobin (Pauling &amp; Coryell 1936). Kebergantungan isyarat resonans magnet kepada aras kepekatan oksigen darah atau BOLD ini telah dilaporkan dalam beberapa kajian awal(Ogawa et al. 1990a, Ogawa et al. 1990b).</p>\r\n		<p>\r\n			Merujuk kepada Jadual 1, Jadual 2 dan Rajah 2, kewujudan ketaksimetrian peng&shy;aktifan antara hemisfera kanan dan kiri, dalam konteks ketinggian isyarat dan ke&shy;luasan kawasan pengaktifan sebagai res&shy;pons terhadap stimulasi yang diberikan menunjukkan bahawa sifat pengkhususan kefungsian juga wujud didalam korteks auditori. Akan tetapi, per&shy;bezaan yang di&shy;tunjukkan dalam nilai pu&shy;rata PPI tersebut adalah tidak bererti (<em>p</em>&gt;0.05). Walau ba&shy;gaimanapun, kepu&shy;tusan kajian ini disokong oleh hasil kajian terdahulu (Loring et al. 1990), yang me&shy;nyatakan bahawa lobus temporal kanan otak manusia memberikan respons yang lebih baik terhadap bunyi bukan pertu&shy;turan. Kajian pendengaran dikotik ke atas subjek normal (Kimura 1964) pula men&shy;dapati bahawa keutamaan hemisfera kiri adalah untuk memproses pertuturan ter&shy;utamanya bunyi konsonan manakala hemisfera kanan berperanan untuk memproses bunyi persekitaran. Dalam satu kajian lain (Peretz 1990) ke atas pesakit angin ahmar, dida&shy;pati bahawa pesakit yang mempunyai lesi pada girus temporal superior hadapan (pSTG) kiri, termasuk korteks auditori, menunjukkan kecacatan dalam persepsi dan pemaha&shy;m&shy;an pertuturan. Akan tetapi, pesakit yang mempunyai lesi pada lobus tempo&shy;ral kanan unilateral tidak menunjuk&shy;kan corak kecacatan persepsi dan pema&shy;ha&shy;man per&shy;tuturan yang sama malah menunjukkan kecacatan dalam persepsi terhadap bunyi bukan pertu&shy;turan misal&shy;nya musik dan bunyi perseki&shy;taran. Hasil kajian terdahulu yang di&shy;nyatakan di atas seka&shy;ligus mene&shy;rangkan mengapa kea&shy;matan maksimum bagi setiap subjek (Rajah 2) terletak pada hemisfera kanan otak.</p>\r\n		<p>\r\n			PPI resonans magnet boleh juga ditentu&shy;kan untuk voksel tertentu misalnya yang menunjukkan keamatan tinggi atau mak&shy;simum dalam suatu kelompok. Mak&shy;lumat yang diperolehi boleh digunakan dalam penentuan kedudukan suatu pusat pem&shy;prosesan maklumat pada otak serta sifat dinamik pusat pemprosesan tersebut yang telah dikenalpasti koordinatnya. PPI bagi voksel berkeamatan tinggi dalam suatu kelompok didapati mem&shy;pengaruhi PPI kelompok tersebut seper&shy;timana yang dapat dilihat dalam Rajah 3.</p>\r\n		<p>\r\n			Daripada Jadual 1, Jadual 2 dan Rajah 2 juga, didapati corak pengaktifan otak yang diperolehi daripada individu ke indi&shy;vidu tidak sama dari segi keluasan kawa&shy;san pengaktifan dan ketinggian isyarat BOLD, walaupun kesemua subjek menun&shy;jukkan pengaktifan pada BA22, 41 dan 42. Perbezaan di antara keputusan yang diperolehi untuk subjek individu je&shy;las menunjukkan kesan spesifik dalam subjek individu yang sememangnya tidak sama antara subjek ke subjek disebab&shy;kan wujud perbezaan intrinsik di dalam setiap subjek. Ini berlaku disebabkan oleh perbezaan dalam kesensitifan otak subjek dalam memberi respons kepada stimulus yang dikenakan memandang&shy;kan isyarat vasodi&shy;latori, aliran darah sere&shy;brum (CBF), isipadu darah serebrum (CBV) dan kuantiti oksi&shy;hemoglobin yang mengawal ketinggian isyarat dan keluas&shy;an kawasan pengaktifan di dalam otak adalah berbeza dalam se&shy;mua individu. Variabiliti ini dianggap seba&shy;gai bersifat intrinsik memandangkan lang&shy;kah untuk mengurangkan artifak pengaktif&shy;an dari&shy;pada faktor pengeliru telahpun diambil supaya ujikaji fMRI yang dilakukan ke atas kesemua subjek adalah sama. Punca lain yang boleh mengakibatkan variabiliti ini adalah ketakseragaman sub&shy;jek dalam menumpukan perhatian kepada apa yang didengarinya walaupun pene&shy;rangan telahpun diberikan kepada setiap subjek sebelum imbasan MRI di&shy;lakukan. Imbasan fMRI yang mengambil masa satu jam keseluruhannya boleh menyebabkan penumpuan subjek ter&shy;ganggu. Pengaruh gerakan subjek ke atas artifak pengaktifan tidak mungkin wujud memandangkan ke&shy;san yang ber&shy;kait dengan gerakan subjek, khususnya secara translasi (dalam arah <em>x</em>, <em>y</em> dan <em>z</em>) dan putaran (dalam arah jong&shy;kang-jong&shy;kit, olengan dan gulingan) telah&shy;pun dikeluarkan semasa proses penjanaan pengaktifan otak menggunakan SPM.</p>\r\n		<p>\r\n			Daripada nilai <em>t</em> yang ditunjukkan dalam Jadual 3 dan 4, iaitu hasil daripada FFX dan RFX, dapat dikatakan bahawa ber&shy;laku pengaktifan otak dengan keamatan yang tinggi dalam kesemua subjek sama ada pada kawasan BA22, 41 atau 42. Stimulus bunyi hingar yang diberi secara binaural dapat merangsang pengaktifan korteks auditori pada kedua-dua belah lobus tem&shy;poral otak. Stimulus bunyi hi&shy;ngar putih adalah stimulus bukan pertu&shy;turan dan ter&shy;bukti dapat diproses dengan lebih baik oleh korteks auditori pada lobus temporal kanan. BA41 dan 42 adalah masing-masing bahagian anterior dan posterior girus Heschl (HG) yang juga dinamakan korteks auditori primer; manakala BA22 pula adalah korteks auditori sekunder pada girus temporal superior (STG) yang dina&shy;makan kawa&shy;san Wernicke. Ketiga-tiga ka&shy;wasan ini terlibat dalam kefungsian audi&shy;tori. Peng&shy;aktifan berlaku secara tak simetri pada kedua-dua hemisfera otak.</p>\r\n		<p>\r\n			FFX adalah analisis kumpulan pering&shy;kat pertama yang dilakukan ke atas ke&shy;san yang wujud dalam kumpulan subjek yang dipilih sahaja. Purata respons yang dicerap dikawal oleh variabiliti dalam setiap im&shy;basan dengan menganggap bahawa setiap imbasan tidak bersandar antara satu sama lain, iaitu &lsquo;kesan malar&rsquo;. FFX dilakukan dengan menggabungkan kesemua im&shy;basan yang diperolehi dari&shy;pada kesemua subjek. Pengaktifan yang ditunjukkan adalah kesan purata dalam kumpulan subjek tersebut dengan vari&shy;ans yang ter&shy;kandung diambil dari im&shy;basan ke imbasan. Ini bermaksud, FFX melibatkan 300 im&shy;basan daripada kese&shy;mua subjek (150 un&shy;tuk keadaan aktif dan 150 untuk keadaan rehat). Oleh yang demikian, inferens yang dibuat hanya sah untuk kumpulan subjek tertentu sa&shy;haja dan tidak boleh digunakan ke atas keseluruhan populasi. RFX pula adalah analisis kumpulan peringkat kedua yang mengambil kira variabiliti dalam analisis kumpulan peringkat pertama dan vari&shy;abiliti yang wujud di antara subjek de&shy;ngan menganggap bahawa subjek telah dipilih secara rawak daripada suatu popu&shy;lasi, iaitu &lsquo;kesan rawak&rsquo;. Oleh itu, jika bi&shy;langan subjek dalam suatu kajian mencu&shy;kupi, inferens yang dibuat melalui RFX ke atas sekumpulan subjek boleh mewakili keseluruhan populasi. Dapat diperhatikan bahawa kawasan pengaktif&shy;an yang terhasil melalui FFX adalah lebih luas dan lebih tinggi keamatannya dari&shy;pada kawasan pengaktifan RFX. Varians dalam FFX diberi oleh <em>s</em><sub>w</sub><sup>2</sup>/<em>Nn</em> dengan <em>s</em><sub>w</sub>=sisihan pia&shy;wai dalam kumpulan sub&shy;jek, <em>N</em>=bi&shy;langan subjek dalam kumpulan dan <em>n</em>=bilangan imbasan bagi setiap subjek. Bagi RFX, varians diberi oleh <em>s</em><sub>b</sub><sup>2</sup>/<em>N</em> + <em>s</em><sub>w</sub><sup>2</sup>/<em>Nn</em> dengan <em>s</em><sub>b</sub>=sisihan piawai antara subjek. Ini ber&shy;makna, nilai <em>t </em>yang ditak&shy;rifkan seba&shy;gai nis&shy;bah magnitud pu&shy;rata pengaktifan terhadap varians bagi pengak&shy;tifan adalah lebih kecil bagi RFX berban&shy;ding FFX. Oleh itu, pada aras keertian yang sama, <em>&alpha;</em><sub>FWE</sub>=0.05, SPMs RFX memberikan jumlah vok&shy;sel aktif ber&shy;erti yang lebih kecil dengan keamatan yang lebih rendah atau mungkin tiada pengaktif&shy;an langsung. Untuk menda&shy;pat&shy;kan pengak&shy;tifan pada SPMs RFX, de&shy;ngan se&shy;bab nilai<em>t</em> nya yang rendah bagi setiap voksel, aras keertian perlu ditu&shy;runkan mi&shy;salnya <em>&alpha;</em>=0.01 (tidak diperbetul&shy;kan).</p>\r\n		<p>\r\n			Daripada analisis FFX, dapat disimpul&shy;kan bahawa respons purata kumpulan menun&shy;jukkan pengaktifan bererti. Walau bagai&shy;manapun, ini tidak bermakna ba&shy;hawa res&shy;pons setiap subjek dalam kum&shy;pulan tersebut bererti atau menunjukkan pengak&shy;tifan yang tekal. Maka, FFX tidak sesuai untuk membuat inferens ke atas popu&shy;lasi, tetapi cukup kukuh untuk mem&shy;buat infe&shy;rens mengenai kumpulan subjek yang ter&shy;libat. RFX walau bagaimanapun menge&shy;sahkan keputusan FFX tersebut. Daripada FFX dan RFX, didapati kedua-dua titik dengan keamatan maksimum berada pada lobus temporal kanan iaitu pada koordinat (56, -20, 10) dan (58, -24, 12). Perbezaan (dalam mm) di antara kedua-dua titik tersebut yang tidak mele&shy;bihi 10 mm menunjukkan ketekalan antara keputusan FFX dan RFX. Dalam kajian ini, pengaktif&shy;an bilateral dapat dikesan pada kedua-dua lobus temporal otak. FFX menunjukkan kawasan peng&shy;aktifan yang lebih luas pada lobus tem&shy;poral kanan. Ini bertentangan dengan RFX. Namun, kawasan yang menunjuk&shy;kan pengaktifan tertinggi dari&shy;pada FFX dan RFX adalah pada BA41 kanan. Keputusan ini tekal dengan kajian terda&shy;hulu yang menyatakan BA41 iaitu se&shy;ba&shy;hagian daripada HG adalah kawasan pemprosesan maklumat auditori utama (Strainer et al. 1997). Selain itu, pe&shy;nyataan mengenai kedominanan otak kanan untuk memproses stimulus audi&shy;tori bukan pertu&shy;turan(Darwin 1971) ter&shy;nyata benar untuk kumpulan subjek dalam kajian ini.</p>\r\n		<p>\r\n			Bagi menyokong keputusan di atas, analisis konjunksi dilakukan. Analisis kon&shy;junksi menggunakan inferens statistik klasik ke atas data bagi mendapatkan ka&shy;wasan pengaktifan sepunya dalam satu kumpulan subjek. Dalam RFX, hipotesis nol yang menyatakan tiada pengaktifan diuji ke atas setiap subjek. Keputusan yang diperolehi boleh digunakan untuk membuat inferens ke atas populasi jika kuasa sta&shy;tistik bagi kajian (dipengaruhi oleh kesan saiz, aras keertian (&alpha;) dan saiz sampel) adalah tinggi, misalnya 80% (Desmond &amp; Glover 2002). Dalam ana&shy;lisis konjunksi, inferens yang lebih sensi&shy;tif mengenai ke&shy;san sepunya digunakan. Walau bagai&shy;manapun, inferens ke atas data yang dibuat hanya sah untuk subjek yang terlibat sahaja dan tidak sah untuk keseluruhan populasi. Perbezaan pada kedua-dua SPMs dalam Rajah 5 adalah disebabkan oleh penggunaan aras ke&shy;yakinan yang berbeza. Dengan meng&shy;gunakan <em>&alpha;</em><sub>FWE</sub>=0.05, nilai <em>p</em> untuk setiap voksel telah diperbetulkan bagi menga&shy;tasi masalah perbandingan berbilang yang wujud dalam pengimejan sains neuro khususnya di antara voksel dan voksel jiran yang saling mempengaruhi. Aras keertian yang dike&shy;nakan dalam analisis jenis ini adalah lebih tinggi ber&shy;banding dengan aras keertian 0.001 menyebabkan bilangan voksel yang menunjukkan pengaktifan bererti menjadi lebih kecil. Daripada hasil analisis kon&shy;junksi ini, sekurang-kurangnya terdapat 5 voksel aktif pada BA41 kanan pada setiap subjek. Jika aras keertian ditu&shy;runkan, mi&shy;salnya menggunakan <em>&alpha;</em>=0.001, kawasan pengaktifan sepunya juga di&shy;tunjukkan oleh kesemua subjek pada BA22 kiri. Kepu&shy;tusan analisis kon&shy;junksi adalah wajar ker&shy;ana tekal dengan kepu&shy;tusan PPI, FFX dan RFX. Tambah&shy;an pula, BA41 pada girus Heschl adalah kawasan otak yang terlibat dalam pem&shy;prosesan maklumat auditori ter&shy;utamanya dalam memberi respons kepada stimulus bukan pertuturan (Penhune et al. 1996, Hart et al. 2002, Griffiths et al. 2007).</p>\r\n		<p>\r\n			<strong>KESIMPULAN</strong></p>\r\n		<p>\r\n			fMRI telah terbukti dapat mengesan per&shy;ubahan dalam respons hemodinamik otak manusia yang berpunca daripada tindak&shy;balas otak terhadap stimulus yang diberi&shy;kan. Data kefungsian otak yang diperolehi daripada imbasan fMRI se&shy;terusnya dapat dianalisis menggunakan SPM. Kelebihan SPM pula adalah kebo&shy;lehannya memberi&shy;kan maklumat menge&shy;nai lokaliti pengaktif&shy;an, bilangan voksel aktif dan keamatan pengaktifan otak. Hasil FFX adalah SPMs yang menunjuk&shy;kan pengaktifan purata subjek yang terli&shy;bat manakala hasil RFX adalah SPMs yang dapat mewakili popu&shy;lasi dari mana subjek dipilih. Kawasan pengaktifan sepunya bagi kesemua subjek yang terli&shy;bat pula telah dapat ditentukan melalui analisis konjunksi.&nbsp; Dapat disimpul&shy;kan bahawa semasa proses pendengaran, terdapat pengaktifan bilateral yang ber&shy;erti (<em>p</em><sub>FWE</sub>&lt;0.05) pada lobus temporal yang tertumpu khususnya pada kawasan BA41 dan BA42 iaitu korteks auditori primer serta BA22 iaitu korteks auditori sekunder. Stimulus bukan pertuturan yang diberikan meyebabkan pengaktifan lebih tertumpu pada hemisfera sebelah kanan berdasar&shy;kan keputusan PPI yang menunjukkan ba&shy;hawa isyarat voksel tertinggi dikesan pada korteks auditori di BA41 kanan serta purata bilangan voksel yang aktif kesemua subjek, iaitu seba&shy;nyak 599&plusmn;1 voksel pada lobus temporal kanan dan 485&plusmn;1 voksel pada lobus temporal kiri. Semua subjek menun&shy;juk&shy;kan pengaktifan bererti (<em>p</em><sub>FWE</sub>&lt;0.05 FWE) pada BA41 kanan semasa proses pendengaran, sekurang-kurangnya seba&shy;nyak 5 voksel. Kajian lanjutan ke atas bi&shy;langan subjek yang lebih ramai diperlu&shy;kan untuk mentahkikkan lagi hasil kajian ini memandangkan keputusan kajian meng&shy;gunakan teknik fMRI (Langers et al. 2003) dan kaedah elektroensefalografi (EEG) (Gour&eacute;vitch et al. 2008) menda&shy;pati bahawa tidak wujud ketaksimetrian pengaktifan otak yang bererti apabila stimulus bukan pertuturan digunakan dalam paradigma ujikaji. Tambahan pula, kedominanan pengaktifan otak pada hemisfera kiri akibat bunyi bukan pertu&shy;turan telah juga ditemui dalam beberapa kajian lain (Celsis et al. 1999).</p>\r\n		<p>\r\n			<strong>PENGHARGAAN</strong></p>\r\n		<p>\r\n			Penghargaan ditujukan kepada Nur Izzati Mat Yusoff dan Sa&rsquo;don Samian di atas bantuan menjalankan ujian pendengaran dan imbasan fMRI ke atas subjek. Kajian ini dibiayai oleh geran Universiti Penye-lidikan GUP-UKM-SK-07-20-205, Universiti Kebangsaan Malaysia.</p>\r\n	</div>\r\n</div>\r\n', 1),
(423, 420, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Schizophrenia is the most difficult mental illness to define and describe (Gelder et al. 2000). The course of illness can be poor and life long treatment is needed. An early observation by Kreaplin (Henry &amp; Smeltzer 2003) noted that 2.5% of pa&shy;tients with schizophrenia recover fully but more recent studies (Kane et al. 1988) have been more optimistic quoting 25% full recovery. The same study also found that 10 -15% of patients with schizophre&shy;nia do not recover fully. For this group of patients, a number of them do not re&shy;spond to standard treatment using single anti-psychotic regime and have been coined treatment resistant. Patients who are resistant to treatment have only been shown to respond to clozapine. Cloza&shy;pine however is not without problems, among them agranulocytosis can occurs, which is a fatal complication that may lead to death if not detected and treated early. Keeping this fact in mind, is there a role for polypharmacy?</p>\r\n<p>\r\n	This case will be used to highlight the rare instances when polypharmacy may be useful prior to the commencement of clozapine for the treatment of schizo&shy;phrenia.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 19 years old college student of Chi&shy;nese descend presented to our center with a week history of aggression and irritability. She was also noted to be talk&shy;ative and there was a history suggestive of depressive illness whereby she was keeping to herself, crying at times and had poor concentration a year before. She however had strong persecutory delusions of being victimized by the pub&shy;lic and kept saying that she was publicly discriminated against. She also claimed to be a special being and able to com&shy;municate with her boy friend telepa&shy;thically whom she blamed to be the rea&shy;son for this. Mentally, she was hostile, but her mood was happy. However her affect was inappropriate, she had flighty ideas and pressure of speech. Her in&shy;sight was poor. She was initially treated for bipolar affective disorder with quetia&shy;pine 400mg twice daily and a mood sta&shy;bilizer, and was discharged 15 days later. She presented the next day with relapse of symptoms when she had sud&shy;denly hit her father in the car and almost caused a motor vehicle accident. During this admission her medication was in&shy;creased and she was discharged 9 days later. She came for one follow up visit but was again admitted two weeks later with relapsing symptoms. Her case was dis&shy;cussed in the postgraduate conference and a revised diagnosis of schizophrenia was made. On this admission, her psy&shy;chotic symptoms were more prominent with persecutory delusions of discrimina&shy;tion, thought broadcasting, neologism, inappropriate affect and intermittent hos&shy;tility. Her mood symptoms had all but disappeared. She received electrocon&shy;vulsive therapy during this admission and aripiprazole at 10mg daily was started. Quetiapine was planned to be tapered off slowly. She was discharged 14 days later with this combination. Dur&shy;ing her admis&shy;sions her biochemical in&shy;vestigations were normal, these included full blood test, renal and liver function test and fasting glucose levels.</p>\r\n<p>\r\n	She was followed up in the clinic and was compliant to both medication and follow up. The quetipaine was reduced gradually and her aripiprazole was in&shy;creased to 15mg once daily. She re&shy;sponded well to this new regimen until she was on aripiprazole 15mg once daily and quetiapine 200mg at night. A re&shy;duction of quetiapine would bring back the delusions and any increase would make her to become too sedated.</p>\r\n<p>\r\n	She has since been maintained on this dose and has responded very well. She plans to continue her studies for which she had stopped for eight months. Her family is also very happy with her progress.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	For the pharmacological treatment of schizophrenia, monotherapy has always been encouraged and advocated in most guidelines(Weiden &amp; Casey 1999, APA 2004). However, polypharmacy has been recognized by some and usually sug&shy;gested as the last resort(Faries et al. 2005). Among reasons why monotherapy is preferred are to allow psychiatrist to accurately evaluate patient&rsquo;s response to the new treatment(Weiden &amp; Casey 1999) to allow adequate trial of each medication, to reduce the complexity of the medication regimen, to reduce side effects and also to assess and manage future symptom exacerba&shy;tions(Miller &amp; Craig 2002).</p>\r\n<p>\r\n	The literature has very little evidence supporting the use of polypharmacy with atypical antipsychotics and is divided on its long-term use this way(Stahl &amp; Grady 2004, Picker et al 2008). Although poly&shy;pharmacy has been used for a variety of reasons with the favorite being to bolster medication effectiveness in treating pa&shy;tients with refractory psychotic symp&shy;toms, mood symptoms or behavioral problems, the risk of combining two atypical antipsychotic appears to out&shy;weigh the benefits (Stahl &amp; Grady 2004). Among these risk is a potentially higher mortality and an increase in metabolic problems(Faries et al. 2005, Stahl 2004).</p>\r\n<p>\r\n	For our patient, polypharmacy appears to work well for her. It has given her an option to recover fully. It has also saved her the option of treatment with clozapine which was impending in view of her poor response to quetiapine and aripiprazole at high doses individually. Although clo&shy;zapine has been proven for difficult cases like our patient, the side effects and regular blood monitoring may be cumbersome given other options in treatment. The use of aripiprazole which is claimed to be weight neutral and less sedating (Lieberman 2004) is better suited for a young and active individual and will hopefully encourage compliance. Although the combination of aripiprazole and quetiapine has not been studied in detail, her low dose of quetiapine at night may be useful for sleep as it is more se&shy;dating but may not be sufficient to cause weight gain as a side effect.</p>\r\n<p>\r\n	Another explanation which may explain her response to this combination could well be that she had improved on the dose of aripiprazole 15mg which she has been taking for more than 3 months. The relapse of symptoms when quetiapine was titrated down and when ariprazole was increased could well be due to aka&shy;thesia, or inner restlessness, which is a known side effect in the initial stages. It can be very discomforting to the patient and may lead her to have a persecutory type of delusion.</p>\r\n<p>\r\n	Whichever the explanation, in the end it is all about the patient and her recovery that matters most.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(424, 421, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	World health and scientific organisations have endorsed breastfeeding as the best way of feeding infants. Breast milk lowers the incidence of gastrointestinal tract problems, infections, allergies and over&shy;weight in the breastfed infants. There is also a decreased risk of death from sud&shy;den infant syndrome. Besides that, mothers who breastfeed have a lower risk of ovarian, uterine and breast cancer, better child spacing and maternal at&shy;tachment with their newborn (Malrides 1995)&nbsp;</p>\r\n<p>\r\n	Malaysian culture traditionally accepted breastfeeding as the normal and healthy way of nourishing a newborn child. With the introduction of Western culture, how&shy;ever, bottle-feeding was introduced and is now viewed as a &ldquo;modern&rdquo; mother&rsquo;s way of providing nutrition to infants (Kang 1999). The National Health and Morbidity Study in 2006, reported a high proportion of Malaysian mothers initiating breast&shy;feeding but the practice of exclusive breastfeeding for four to six months was low (14.5%) (Ministry of Health 2006). The prevalence of breastfeeding in Ma&shy;laysia declined from 92% to 78% from 1950 to 1975 however it increased to 89% by 1996 (Ho et al. 1999). A study done in 1996 among Malay mothers in Kelantan found that almost all mothers (97.3%) breastfed their children, indicat&shy;ing a high incidence of breastfeeding among rural Malay mothers (Zulkifli et al. 1996). The current study was designed to determine the practice of initiating breastfeeding among mothers in an urban community, its associ&shy;ated factors, and evaluate the mothers&rsquo; knowledge regarding breastfeeding.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This was a cross-sectional study con&shy;ducted among mothers attending a Child Health Clinic in Cheras Baru, Kuala Lumpur between March and April 2008.&nbsp; All mothers who attended the clinic dur&shy;ing the study period who fit the selection criteria were included into the study. In&shy;clusion criteria were mothers aged from 17 to 45 years old who gave consent to answer the questionnaire. Exclusion cri&shy;teria were primigravida (no experience in breast feeding) and non-Malaysian.</p>\r\n<p>\r\n	Respondents were given a self-admin&shy;is&shy;tered questionnaire and were assisted by interviewers when necessary. Respon&shy;dents&rsquo; data were collected via question&shy;naire. The questionnaire consist-ed of two parts. The first part was on socio-demo&shy;graphic data and the second part was on breastfeeding knowledge.</p>\r\n<p>\r\n	Successful initiation of breastfeeding was defined as mothers who breastfed their child for at least one week after deliv&shy;ery (Khairani 1998). &ldquo;Significant family members&rdquo; was defined as respon&shy;dent&rsquo;s spouse and family members who stayed in or close by her residence.</p>\r\n<p>\r\n	All data were entered and analyzed using the Statistical Analysis for Social Science (SPSS) windows, version 14.0. A p value of less than 0.05 was taken to denote statistical significance.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 162 mothers were included in the study. It was found that 152 mothers practiced breastfeeding at least one week after delivery giving a prevalence of 93.8%. The mother&rsquo;s characteristics are summarized in Table 1.</p>\r\n<p>\r\n	Mothers who practise breastfeeding were predominantly Malay. Influence from significant family members was found to be an important factor towards encouragement of breastfeeding. In this study, mother&rsquo;s age, education and em&shy;ployment status, husband&rsquo;s occupation and family income did not show any sig&shy;nificant association with breastfeeding.</p>\r\n<p>\r\n	Figure 1 shows the source of knowl&shy;edge about breastfeeding among the re&shy;spondents. The top three important sources found in this study were health&shy;care personnel (25.0%), mass media (books and magazines) (21.2%) and par&shy;ents or relatives (18.5%).</p>\r\n<p>\r\n	Table 2 shows the relationship between antenatal class and breastfeeding prac&shy;tice. More than half of the respondents (55.6%) attended antenatal education about breastfeeding when they were pregnant. In the breastfeeding group, 85 respondents (55.9%) had formal ante&shy;natal education on breastfeeding. How&shy;ever, attending antenatal classes did not significantly increase the practice of breastfeeding.</p>\r\n<p>\r\n	Table 3 illustrates the assessment of knowledge on breastfeeding among the mothers. Overall, the mothers&rsquo; know&shy;ledge on breastfeeding was fairly good. However, more than half of the mothers thought that &lsquo;they should stop breast&shy;feeding if either or both of baby and mother is/are sick.&rsquo; More than 20% of the mothers were still ignorant regarding lactational amenorrhoea, breast milk protection against allergy and the preva&shy;lence of sore nipples. Approximately 15% of the mothers did not know that breast milk is better than formula milk and about 9% were not knowledgeable regarding the benefits of colostrum.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The prevalence of breastfeeding in this study was 93.8%. The prevalence was comparatively similar to previous studies done in Malaysia (Ho et al. 1999; Zulkifli et al. 1996). The high prevalence of breastfeeding is viewed as a result of greater awareness of the benefits to health and nutrition for breast fed infants.&nbsp; However, this study did not assess if the mothers continued breast&shy;feeding beyond a week. It had been re&shy;ported that a high proportion of Malay&shy;sian mothers initiated breastfeeding but the practice of exclu-sive breastfeeding for four to six months was low (Ministry of Health 2006).</p>\r\n<p>\r\n	In this study, Malay mothers were more likely to breastfeed than non-Malay. The result is supported by The National Health and Morbidity study done in Ma&shy;laysia (Ministry of Health 2006). In the Malay culture and Islamic religion, moth&shy;ers are encouraged to breastfeed their children and this could explain the high breastfeeding prevalence in this ethnic group (Counsilman &amp; Viegas 1985).</p>\r\n<p>\r\n	The findings in this study showed that a woman&rsquo;s partner and family play an im&shy;portant role in her decision to breast&shy;feed. This finding is supported by west&shy;ern studies which reported positive as&shy;socia&shy;tion between partner&rsquo;s support for breastfeeding with initiation as well as duration of breastfeeding (Scott et al. 2001). Thus, significant family encour&shy;agement is a positive pre&shy;dictor for breastfeeding practice.</p>\r\n<p>\r\n	Previous studies showed that educated women were more likely to breastfeed their children (Simard et al. 2005; Lin et al. 2004). The father&rsquo;s education level, too, has an enhancing effect on breast&shy;feeding (Newton 2004). However some studies found the oppo&shy;site (Mohammad et al. 2006; Li et al. 2002). Likewise our finding did not support these relation&shy;ships. The prevalence of initiating breastfeeding had no significant associa&shy;tion with mothers&rsquo; employment status, husband&rsquo;s occupation or family income.</p>\r\n<p>\r\n	Antenatal class is the medium for pro&shy;viding information which includes promo&shy;tion of breastfeeding. Systematic reviews conclude that educational programs are more effective at improving breastfeeding initiation (Dyson et al. 2005; Guise et al. 2003) than literature alone (Fairbank et al. 2000).&nbsp; However, previous studies had reported that participation in antenatal class had no significant association with prevalence of initiating breastfeeding (Citra et al. 2007; Foo et al. 2005). A possible explanation for this could be that those who did not attend received sup&shy;port and knowledge from other sources.</p>\r\n<p>\r\n	With regard to knowledge on breast&shy;feeding, the majority of respondents knew that breastfeeding should be initi&shy;ated immediately after birth and exclusive breastfeeding should be practised for the first six months. This could be due to the successful breastfeeding promotion practiced in many government and pri&shy;vate hospitals. However it is a concern that approximately 15% of the mothers did not know that breast milk is better than formula milk and about 9% were not knowledgeable regarding the benefits of colostrums. Almost half of the mothers thought that breastfeeding should be dis&shy;continued when either mother or child is sick.&nbsp; This misconception should be cor&shy;rected by health providers during the breastfeeding educational programmes.&nbsp; In addition, more than 20% of mothers were still ignorant regarding lactational amenorrhoea, protection against allergy and that sore nipple is a common prob&shy;lem when breastfeeding. These are some areas of breastfeeding that need to be addressed and given more emphasis to mothers to improve their knowledge of breastfeeding.</p>\r\n<p>\r\n	Overall, the mothers&rsquo; knowledge on breastfeeding was fairly good except for certain areas mentioned above.&nbsp; This may reflect successful antenatal pro&shy;grammes initiated by our society. It could also be explained by the great effort from health personnel in delivering talks on the benefits of breastfeeding. This is shown by the findings in this study, where the top three sources of knowl&shy;edge about breast feeding indicated by the mothers were healthcare personnel, mass media (books and magazines) and parents. These main sources of know&shy;ledge can be used as a channel to cor&shy;rect&nbsp;&nbsp; mis&shy;conceptions and improve moth&shy;ers&rsquo; knowledge on breastfeeding.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The prevalence of initiating breastfeeding among mothers attending the Maternal and Child Health clinic was high. Majority of the mothers had adequate knowledge regarding breastfeeding, however certain areas such as &lsquo;continuing breastfeeding when either mother or child is sick&rsquo;, lac&shy;tational amenorrhoea, protection against allergy and &lsquo;sore nipple is a common problem when breastfeeding&rsquo; need more emphasis during future educational pro&shy;grammes. Influence from close family members was significantly associated with breastfeeding practice. Thus, it would be beneficial to include close fam&shy;ily members during health education pro&shy;grammes to promote breastfeeding.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	We would like to express our sincere ap&shy;preciation to Mr. Nik Muhammad Shahid Nik Jaffar, Mr. Mohd Khalid Al Walid Abas, Miss Nurul Azreen Yusof, Miss Nur Hairi Nahar, Miss Rohayu Abdul Halim for their assistance.</p>\r\n', 1),
(425, 422, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Alzheimers disease (AD) is characterized by regional neurodegeneration, synaptic loss and the presence of senile plaques (SantaCruz et al. 2005). The senile plaques consist of abnormal interneu-ronal growth, dead cells and beta amyloid. There has been increasing evi&shy;dence which suggests that the buildup and ag&shy;gregation of beta amyloid peptide (A&beta;) in the brain plays a primary role in the pa&shy;thogenesis of AD (Piccini et al. 2005).</p>\r\n<p>\r\n	&nbsp;Although A&beta; has been demonstrated to be neurotoxic in cell culture (Butterfield et al. 2004), the mechanism by which it ex&shy;erts its toxicity is still unclear. Several mechanisms have been suggested in&shy;cluding increased sensitivity to excito&shy;toxins, alterations to calcium homeosta&shy;sis (Celsi et al. 2006), activation of re&shy;ceptor that precipitates cell destruction and activation of inflammatory pathways (Zou et al<em>.</em> 2006).&nbsp;</p>\r\n<p>\r\n	&nbsp;Recently, the involvement of oxidative stress in the pathogenesis of AD has been suggested (Wang et al. 2008). The AD brain was reported to have increased lipid peroxidation, increased carbonyl modification of proteins and increased oxidation of mitochondrial DNA (Arimura&amp; Kaibuchi 2005).</p>\r\n<p>\r\n	&nbsp;Increase oxidative stress occurs when the balance between free radical genera&shy;tion and antioxidant capacities shifts to&shy;ward free radical generation leading to oxidative damage to lipids, protein, RNA and DNA. This is not surprising as the brain is more susceptible to oxidative damage compared to other organs or tissues, due to its high rate of oxygen consumption, high polyunsaturated lipid content, and relative paucity of antioxi&shy;dant enzymes (Puttfarcken 1996).</p>\r\n<p>\r\n	Understanding how A&beta; acts and its probable association to oxidative stress is important for the formulation of treat&shy;ment and preventive steps against neu&shy;rodegeneration. Therefore, in this study, the involvement of oxidative damage in the mechanism of A&beta;-toxicity resulting in neuronal cell death was determined by measuring oxidative DNA damage. The involvement of oxidative damage was further elucidated by determining the ef&shy;fect of TRF.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>SH-SY5Y neuroblastoma cell culture</em></p>\r\n<p>\r\n	Human neuroblastoma SH-SY5Y cells were gifts from Dr. Coral Sanfeliu of the Institut d&rsquo;Investigacions Biom&egrave;diques de Barcelona, Spain. Cells were grown un&shy;der the following condition: 50% of EMEM with Earles Salt pH 7.2 (Flowlab, Australia) and 50% Ham&#39;s F-12 pH 7.2 (Sigma, USA) supplemented with 200mM Non-essential amino acid (NEAA) (Flowlab, Australia), 10mg/ml gentamicin (PAA, Austria) and 10% Foetal Bovine Serum (FBS), heat inactivated (PAA, Austria). Cells were subcultured after wash with phosphate buffered saline (PBS) and trypsin. Cells were then seeded at 1.5 x 10<sup>4</sup> cells per ml on 96-well plates. Cultures were maintained in 5% CO<sub>2 </sub>/ 95% air at 37<sup>O</sup>C. Experiments were carried out on the 7<sup>th</sup>-9<sup>th</sup> day.</p>\r\n<p>\r\n	<em>Preparation of Aggregated A&beta;</em></p>\r\n<p>\r\n	The stock solution of A&beta;<sub>1-42 </sub>(Calbiochem, German) was dissolved at 1mg/ml in 50Mm Tris-HCL, pH &ge; 9.0. The peptide was diluted to 20&mu;&Mu; with calcium, mag&shy;nesium-free PBS and stored at -20<sup>o</sup>C. The stock solution was then diluted to a final concentration of 10&mu;&Mu; followed by incubation at 37<sup>o</sup>C for 24 hours before use.</p>\r\n<p>\r\n	<em>Experimental Design</em></p>\r\n<p>\r\n	Cells were divided into 4 groups: un&shy;treated cells served as control, treated with 10&mu;M A&szlig; peptide for 24 hours, 5mg/ml TRF for 24 hours followed by 10&mu;M A&szlig; peptide for 24 hours (pre treatment) or 10&mu;M A&szlig; peptide for 24 hours followed by 5mg/ml TRF for 24 hours (post treatment). All cultures were maintained in 5% CO<sub>2 </sub>/ 95% air at 37<sup>o</sup>C. TRF was pur&shy;chased from Golden Hope Biorganic Sdn. Bhd. (Malaysia). DNA damage, cell viability and apoptosis were determined by using MTS assay, comet assay and Propidium Iodide &amp; Calcein-AM staining respec&shy;tively.</p>\r\n<p>\r\n	<em>Comet assay</em></p>\r\n<p>\r\n	DNA damage was determined by using the Comet assay according to the me&shy;thod described by Singh et al. (1988), with minor modifications. 105&mu;l of normal melting agarose (NMA) was added to frosted microscope slides (Curtin Matherson, USA), covered with cover&shy;slips and kept for 10 minutes to solidify. Co&shy;verslips were removed and 20&mu;l cells with 80&mu;l low melting point agarose (LMA) were added to the slides (Sigma, USA). The slides were then immersed in a jar containing cold lysing solution (2.5 M NaCl, 100 mM ethylene-diaminetetra-acetic acid, 10 mM Tris at pH 10, 1% Triton X-100, 1% sodium N-lauroyl sar&shy;cocinate, 10% dimethylsulfoxide)&nbsp; for at least 1 hour. After lysis, the slides were placed in electrophoresis box filled with electrophoresis buffer (0.3 M NaOH and 1 mM ethylene-diaminetetra-acetic acid). The cells were incubated for 20 minutes to allow for unwinding of DNA and ex&shy;pression of alkali-labile sites. All of these steps were conducted under dim light to prevent additional DNA damage. After electrophoresis, neutralization buffer (0.4M Tris, pH 7.5) was added to neu&shy;tralize the alkali, and the slides were left at room temperature for 5 minutes. This step was repeated twice. Ethidium bro&shy;mide (Sigma, USA) was added to each slide, covered with a co&shy;verslip, kept in a humidified box and fi&shy;nally analyzed using a fluorescence mi&shy;croscope (AxioCam MRC, Carl Zeiss, Germany).</p>\r\n<p>\r\n	<em>MTS assay</em></p>\r\n<p>\r\n	The MTS assay purchased from (Promega, USA), employs MTS and the electron coupling agent phenazine me&shy;thosulphate (PMS). MTS is converted into a medium soluble forma&shy;zan product by dehydrogenase enzymes found in metabolically active cells. 20 &mu;l of MTS reagents was pipetted into each well containing the samples in 100&mu;l of cul&shy;ture medium. The plates were then incu&shy;bated for 2 hours in 5% CO<sub>2</sub>/95% air at 37<sup>o</sup>C. The optical density (OD) of the wells was determined using a microplate reader at 490-nm wavelength (Versa max, Japan).</p>\r\n<p>\r\n	<em>Propidium Iodide &amp; Calcein-AM staining</em></p>\r\n<p>\r\n	Staining to detect cell death is based on the principle that live cells have intracel&shy;lular esterase that converts non-fluores&shy;cent cell-permeable calcein-AM to the intensely fluorescent calcein which is re&shy;tained within the cells. Viable cell mem&shy;branes are impermeable to propidium iodide. Dead cells however, allow propi&shy;dium iodide to enter and bind to nucleic acid. Thus live cells stained green with calcein-AM while dead and apoptotic cells stained red with propidium iodide (Sigma, USA). 30&mu;g/ml calcein-AM and 7.5 &mu;g/ml propidium iodide were added to the cell cultures in chamber slides (Nunc, Denmark) and incubated for 30 minutes. Thereafter, cultures were washed with PBS, fixed with fresh 4% paraformalde&shy;hyde and then incubated for 30 minutes at 37<sup>O</sup>C. Slides were washed and cover&shy;slips mounted for microscopic examina&shy;tion (AxioCam MRC, Carl Zeiss, Germany).</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>DNA Damage Studies</em></p>\r\n<p>\r\n	Figure 1 shows DNA damage in the treated and untreated cells as deter&shy;mined by comet assay. Cells were grouped according to the length of comet tail (%). Cells without DNA fragmentation were classified as no DNA damage, cells with DNA fragmentation were classified as having mild damage and cells with major fragmentation as severely dam&shy;aged. Re&shy;sults showed that incubation with 10&mu;M A&szlig; peptide significantly in&shy;creased the DNA damage (p&lt;0.05). However, cells treated with TRF before (pre treatment) or after (post treatment) incubation with A&beta; was observed to have a significantly reduced number of DNA damage. There was no significant differ&shy;ence observed between the number and severity of DNA damage in pre and post TRF treated groups.</p>\r\n<p>\r\n	<em>Determination of A&beta; Peptide toxicity</em></p>\r\n<p>\r\n	The concentration of A&beta; peptide which can induce 50% cell death (IC<sub>50</sub>)was determined by incubating cells with in&shy;creasing concentration of A&beta; peptide. Figure 2 shows that this concentration was 10&mu;M and hence 10&mu;M A&beta; peptide was the concentration used in subse&shy;quent experiments.</p>\r\n<p>\r\n	<em>Neuroprotection&nbsp; Studies</em></p>\r\n<p>\r\n	Figure 3 shows that 10&mu;M A&beta; signifi&shy;cantly increased neuronal death. Pre and post treatment with 5mg/ml TRF signifi&shy;cantly increased the number of viable cells compared to the cells treated with A&beta; only and thus demonstrated that TRF was able to prevent cell death induced by A&szlig; peptide.</p>\r\n<p>\r\n	Figure 4 shows the results of fluores&shy;cence staining using calcein AM and propidium iodide. Control live neuron cells were stained green (a) whereas cells exposed to 10&mu;M A&szlig; peptide were stained red with (i) cell shrinkage and (ii) condensed chromatins, characteristics of apoptotic cells (b). Cells pretreated with 5mg/ml TRF before exposure to A&szlig; un&shy;derwent early apoptosis but still retained membrane integrity (c). TRF post treated cells showed increased number of viable cells compared to cells exposed to A&beta; only demonstrating that post treatment of cells with TRF prevented cell death in&shy;duced by A&szlig; (d).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Recent evidence suggests that A&beta; pep&shy;tide may contribute to the progressive neuronal loss and/or may be directly neu&shy;rotoxic both in vitro and in vivo (Pike et al. 1993). This is consistent with the present finding where A&beta; peptide was observed to significantly decrease the number of viable cells (Figure 2). The induction of cell death may be elicited via apoptosis (Behl et al. 1994). The present finding (Figure 4b) showed that the num&shy;ber of apoptotic cells was increased while the number of viable cells decreased when exposed to A&beta; peptide.</p>\r\n<p>\r\n	&nbsp;A&beta; peptide may exert its cytotoxic ef&shy;fects by activating the apoptotic pathway via free radical damage and/or induction of the signaling molecules (Pereira et al. 1999). Free radicals are constantly gen&shy;erated <em>in vivo</em> and have been reported to be significant contributors to the devel&shy;opment of chronic degenerative disorders such as Alzheimers disease (Ames et al. 1993). Although multiple antioxidant re&shy;pair systems exist to help control this process, reactive species still cause damage to biomolecules, cells and tis&shy;sues.</p>\r\n<p>\r\n	&nbsp;DNA damage is probably the most sig&shy;nificant biological target because this may ultimately lead to altered gene ex&shy;pression, disruption of cellular repair me&shy;chanism and distorted cellular function (Heaton et al. 2002). Thus DNA damage was measured in this study after cells were incubated with A&beta; peptide with and without treatment with TRF.</p>\r\n<p>\r\n	&nbsp;Comet assay was used to measure DNA damage and visual scoring method was applied to evaluate the amount of DNA damage (Collins 2004). The present results showed that DNA damage was significantly increased in cells incubated with A&beta; peptide (Figure 1). This may be attributed to the observation that A&beta; pep&shy;tide could generate ROS which contri&shy;bute to the DNA damage.</p>\r\n<p>\r\n	&nbsp;Treatment with TRF, either before (pre) or after (post-treatment) incubation with A&beta; peptide confirmed the neuroprotective effect of TRF. The data from this study showed that TRF prevented cell death, minimized apoptosis and reduced the DNA damage induced by A&beta; peptide. Many studies have shown that vitamin E and its isomers exert neuroprotective effects against ROS-induced cell death in vitro (Sen et al. 2006; Mazlan et al. 2006). A recent report also demonstrated that AD patients with moderately severe impairment respond favorably to &alpha;-toco&shy;pherol by slowing the progression of the disease (Sano et al. 1997). An epidemi&shy;ological study involving 633 persons over 65 years old suggests that the use of high dose vitamin E supplements may lower the risk of AD (Morris et al. 1998). The neuroprotective effect of TRF may be exerted via its antioxidant effects or by acting at the gene level, affecting the ex&shy;pressions of the signaling molecules of the apoptotic pathway.</p>\r\n<p>\r\n	&nbsp;The results thus show the relationship between the oxidative DNA damage, apoptosis and cell death induced by A&beta; peptide which is prevented by TRF. In conclusion, A&beta; peptide cause DNA dam&shy;age and ultimately cell death via apopto&shy;sis probably by inducing oxidative DNA damage. This is further supported by the fact that TRF, an antioxidant is able to prevent the DNA damage, apoptosis and cell death.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	This study was funded by UKM-FF-03-FRGS0006-2006 research grant.</p>\r\n', 1),
(426, 423, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Drug administration is an integral part of nurses&rsquo; role and is performed in their eve&shy;ryday nursing practice. The practice in drug management includes preparing, checking and administering medications, continuous updating of knowledge in me&shy;dications, monitoring the effectiveness of treatment, identifying side-effects and providing medication education (Shea, 1999). With regards to their diverse roles in providing medication and care, nurses are required to have adequate know&shy;ledge on the therapeutic uses, normal dosage, side-effects, correct route, pre&shy;cautions and contra-indications of drugs. Nurses are therefore, responsible in en&shy;suring that the medications administered according to standard practice as they are accountable for their own practice.</p>\r\n<p>\r\n	Medication error is reported to be a common problem in the clinical settings. Nurses require the essential knowledge on pharmacology, portray a positive atti&shy;tude and competence in medication ad&shy;ministration to prevent any error. Ade&shy;quate knowledge of medication and good practice will definitely assist nurses in administering medications effectively and correctly.</p>\r\n<p>\r\n	Studies have shown that knowledge in pharmacology among the nurses were low and inadequate. (Clancy et al. 2000; Latter et al. 2000; Manias &amp; Bullock, 2002b). Boggs et al. (1998) assessed nurses&rsquo; pharmacology knowledge by testing their knowledge of three com&shy;monly prescribed drugs. The mean score in knowledge on pharmacology was only 46%. Findings showed that nurses lacked sufficient knowledge on dosages, mechanism of action and pharmacoki&shy;netics. The highest knowledge levels were found for clinical indicators for the drugs and side-effects. Findings also showed that medication knowledge tends to increase with educational level but is not related to experiential background.</p>\r\n<p>\r\n	Ivel et al. (1996) also surveyed the ac&shy;tual and self-rated pharmacology know&shy;ledge of nurses. The average test score produced was 56%. There was found to be a relationship between test scores and the number of years of practice. Nurses&rsquo; knowledge on medication was inadequate, especially pertaining to the drug and its side-effects (Alibhai et al. 1999; Henderson &amp; Zernike 2001).</p>\r\n<p>\r\n	Manias and Bullock (2002a) indicated that registered nurses in Australia lacked in-depth knowledge in pharmacology and experience difficulties in understanding and demonstrating pharmacological con&shy;cepts. A number of studies (Leonard &amp; Jonett 1990; Courtenay 1991 and Ives et al. 1996) have found that nurse&rsquo;s know&shy;ledge level on both pharmacology and medical sciences to be inadequate. Gerry &amp; Helen (2003) reported that length of ex&shy;perience and level of professional edu&shy;ca&shy;tion was associated with level of know&shy;ledge in the administration of medica&shy;tions. Having extra educational qualifica&shy;tions among nurses were found to have contributed to a better knowledge of me&shy;dications. Markowitz et al. (1981) found out that nurses working on the day shift had greater knowledge of medication than nurses who worked on night shift.&nbsp; Mattan (1998) revealed that administer&shy;ing medicine effectively was a feature of practice but it lacked quality, which is due to poor knowledge in pharmacology and practice. Manias et al. (2004) indicated that nurses possessed the necessary knowledge and skills in medication ad&shy;ministration such as monitoring effects of medication, assessing and evaluating patient&rsquo;s condition prior to medication administration. However, the nurses were found to have poor attitude as most nurses did not evaluate or monitor pa&shy;tient&rsquo;s condition following the administra&shy;tion of medications.</p>\r\n<p>\r\n	Many medication errors committed were found to be based on pharmaco&shy;logical knowledge, attitude and skills in medica&shy;tion administration. Statistics (1999-2000) cited by Gerry and Helen &nbsp;(2003) has shown that 5.41% of the me&shy;dication er&shy;rors occurred were due to the failure in following the prescription &ndash; wrong dose, wrong route and poor skills in adminis&shy;tering drugs. Gladstone (1995) also iden&shy;tified know&shy;ledge, skills, break&shy;down in communica&shy;tion and failure to follow pol&shy;icy, as the main cause of medi&shy;cation er&shy;ror.</p>\r\n<p>\r\n	Nurses also play an important role in medication education. Mattan (1998) re&shy;vealed that medication education is also a feature in nursing practice but it lacked quality as nurses lacked knowledge and confidence and therefore were unable to provide medication education to patients. Syred (1981) and Elkind (1982) have suggested that nurses fail to contribute to patient education due to the lack of knowledge and skills in pharmacology.</p>\r\n<p>\r\n	To determine whether regis&shy;tered nurses are equipped with the ne&shy;cessary knowledge, skills and confidence to manage their patient&rsquo;s medication, it is therefore crucial to explore and examine their knowledge, attitude and skills. There is no available data in Malaysia regarding nurses&rsquo; knowledge, competence and at&shy;titude in medication administration. The aim of this study was to measure the level of knowledge, skills and practice of nurses in the medical ward in adminis&shy;tering oral medication and whether nurse&rsquo;s knowledge, attitude and practice has any association with working expe&shy;rience and post basic training.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross-sectional descriptive design was used in this study. A sample of 40 regis&shy;tered nurses, working in the medical wards in UKMMC was re&shy;cruited to par&shy;ticipate in this study. Rea&shy;sons for con&shy;ducting this study on nurses working in medical ward was due to the various types of medical drugs used in patients with medical problems and high inci&shy;dence of medication errors reported. UKMMC is a teaching hospital and train&shy;ing centre for nurses and doctors and therefore nurses working in this hospital are required to possess a good level of pharmacological knowledge and good atti&shy;tude in medication administration.</p>\r\n<p>\r\n	A pilot study was conducted on ten nurses working in surgical wards to as&shy;sess the reliability of the questionnaires before the actual study. Nurses who par&shy;ticipated in the pilot study were excluded from the main study.</p>\r\n<p>\r\n	Instruments used in this study were in the form of Checklist and Questionnaires. Checklist was used as the researcher used the direct observation method to assess the level of practice of partici&shy;pants. The checklist consisted of the re&shy;quired activities to be performed in the administration of medication that were derived from the &ldquo;Fundamental of nursing books&rdquo; and redesigned by the research team.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Instrument 1</strong>- Checklist consisted of two sections - demographic data and the medication skills to be performed by nurses. Demographic data con&shy;sisted of years of experience and professional education. Professional education is de&shy;scribed as either Dip&shy;loma or Diploma with Post Basic (Ad&shy;vanced Nursing). A respondent with more than 2 years of working expe&shy;rience is described as expe&shy;rienced &amp; less than 2 years as non-expe&shy;rienced.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Instrument 2</strong>- Questionnaires con&shy;sisted of 2 sections: Section A on pharmacol&shy;ogy knowledge &amp; Section B - questions on attitude of nurses in medication ad&shy;ministration.</p>\r\n<p>\r\n	All data were coded and entered into SPSS (The Statistical Package for Social Sciences) for analysis. An independent t-test was used to determine the influence of working experience and post basic course on knowledge, attitude and prac&shy;tice. Distribution of scores in knowledge, attitude and practice were rated as high/good, average and low/poor. Know&shy;ledge scores &gt;17 were rated as high knowledge, between 11-17 average knowledge, and &lt;11 as having poor knowledge. Attitude scores &gt;18 were deemed as good attitude, scores be&shy;tween 13-18 as average attitude and &lt;13 as poor attitude. Practice scores &gt;12 were deemed as good practice, 7-12 as average and &lt;7 as poor practice. This reference range was developed from the number of questionnaires asked and scores given for each answer.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The characteristics of the 40 respon&shy;dents are presented in Table 1. Eight (8) nurses were found to have an experience of less than two years and majority (n=32) as having experience of more than two years. A total of ten (25%) nurses were found to have post basic courses and 30 (75%) had only Diploma qualification.</p>\r\n<p>\r\n	<em>Nurses skills in medication administration</em></p>\r\n<p>\r\n	Six activities commonly performed in re&shy;lation to medication administration was observed (Table 2) and marked accord&shy;ing to the checklist.</p>\r\n<p>\r\n	Prior to serving medicine, 67.5% prac&shy;ticed hand washing and all nurses (100%) prepared their medication trolley. 77.5% served medication at the correct time and 87.5% practiced correct method of serving oral medication. 50% of the nurses ensured that patients swallowed the medication immediately upon serving, whereby the other 50% was found to have either placed the medication on the patient&rsquo;s locker or gave the medications to the cares. All nurses (100%) docu&shy;mented the medications served.</p>\r\n<p>\r\n	<em>Nurses&rsquo; knowledge in Pharmacology</em></p>\r\n<p>\r\n	Questions related to Pharmacology knowledge as shown in Table 3 was answered by all the nurses. On the ques&shy;tion of which medication administration was considered an oral route, all knew sublingual (100%) was an oral route. Only 27 nurses (67.5%) iden&shy;tified buccal as an oral route. However, topical (100%) and transdermal (5%) was also consi&shy;dered as an oral route by the nurses.</p>\r\n<p>\r\n	In response to questions asked on the serving of special category of medica&shy;tions such as antibiotics, lozenges, GTN and aspirin, 50% of the nurses agreed antibiotics should be served after meals and the others (50%) before meals. On how lozenges should be taken, 55% re&shy;ported that lozenges should be chewed slowly and 45% stated it should be swal&shy;lowed as any other tablets. On whether GTN is taken sublingually, 90% of the nurses gave a correct answer. 52.5% nurses indicated Aspirin is to be crushed and dissolved in water before serving. On the identification of drugs classified under antibiotic, all nurses knew Metronidazole was a fungal antibiotic. However, there were a handful of nurses who identified Methotrexate (7.5%), Metoclopramide hydrochloride (10%) and Methylpredniso&shy;lone (7.5%) as antibiotics, which is incor&shy;rect.</p>\r\n<p>\r\n	All nurses indicated that antibiotics should be served at the correct time.&nbsp; Nurses (100%) also indicated that medi&shy;cation in the liquid form is easily absorb&shy;able compared to tablets. Nurses re&shy;membered the main important five rights (Right Patient, Right Drug, Right Dosage, Right Time and Right Route) in serving medica&shy;tion with ease, which forms the founda&shy;tion for safe practice.</p>\r\n<p>\r\n	<em>Nurses Attitude in Medication Administration</em></p>\r\n<p>\r\n	Ten items as described in Table 4 were used to measure nurses&rsquo; attitude in medi&shy;cation administration. Nurses agreed that pharmacology knowledge should be up&shy;graded from time to time. All agreed it is nurses&rsquo; responsibility to serve medica&shy;tions however busy they are, but more than half of the nurses said they will ask for assistance from their colleagues to help prepare and serve the medications. All nurses indicated, they will not hesitate to ask nurse aids to serve the medica&shy;tions. 100% indicated they will not hesi&shy;tate to place patient&rsquo;s medication on the locker if such a situation arises. However, 100% strongly indicated that they will follow up their patients to ensure that they have taken their medication even after completing their medication round. 65% of nurses indicated that they will leave their medication trolley to answer phone calls. Nearly half of the nurses said they will not mind to administer drugs prepared by others. Nurses were asked on what they will do if they en&shy;counter problem or confusion about a drug; 90% of the nurses indicated that they will get assistance from colleagues, will let it remain as it is (12.5%) and seek pharmacist assistance (100%). Hundred percent of the nurses indicated that hand washing is important prior to serving me&shy;dication.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Medication administration is a daily ba&shy;sic activity in nursing practice and nurses need to have sufficient knowledge, atti&shy;tude and competency to perform these tasks. The findings of this study suggest that nurses do have sufficient pharma&shy;cological knowledge but only in certain aspects of pharmacology.&nbsp; This is clearly reflected in the test scores achieved. These findings are similar to the study done by King (2004) and Coombs et al. (2003) and thus support the need for pharmacology knowledge in practice. Knowledge on drug administration is li&shy;mited. 45% of the respondents did not know that lozenges should be chewed and 33% of the nurses did not know that the buccal route is also linked to an oral method of drug administration, and placed in the buccal region. The term &ldquo;topical&rdquo; means local application to the skin but 100% of the nurses indicated that &ldquo;topical&rdquo; is an oral method.</p>\r\n<p>\r\n	Nurses need to improve their know&shy;ledge especially on the different routes of me&shy;dication administration as responsibil&shy;ity for correct administration of medica&shy;tion lies in the hands of nurses. The pur&shy;pose of giving an antibiotic before meals is for better absorption. Most of the anti&shy;biotics are to be taken before meal time except for the exceptional ones which irritate the mucous membrane of the stomach. However, in this study, 50% of the nurses are ignorant that antibiotics are to be served before meals unless indicated so. GTN is a common drug used in medical wards and nurses have been taught to administer it sublingually, however, it is a surprise, as 10% of nurses who are working in the Medical ward failed to know this. Methotrexate and Methyl&shy;prednisolone has been identi&shy;fied as an antibiotic by 100% of the nurses which is not correct. It indicates a limited know&shy;ledge on &ldquo;action of drugs&rdquo; as nurses did not produce high scores in some of these &ldquo;must know&rdquo; pharmacol&shy;ogy questions. It is important as they are commonly prescribed and have learnt it during their basic training. Low pharma&shy;cology knowledge among nurses can lead to medication errors as safe medi&shy;cation administration is based on know&shy;ledge, attitude and sound clinical judgement. Findings of this study are similar to that of Jeannes and Taylor (1992) and Ashirt (1993), and comments have been made on the limited knowledge of phar&shy;macology among registered nurses. This issue needs to be addressed ur&shy;gently or else patient safety will be af&shy;fected and quality patient care will be compromised.</p>\r\n<p>\r\n	Attitude and practice is somehow said to be co-related in previous studies, but this is not the case here as a flaw was detected (Manias &amp; Bullock 2002a; Ivel et al. 1996). In ensuring patients swallowed the medication immediately, the same question was tested in attitude and practice and 100% nurses agreed that they would stay with patients until medication is swallowed but the test score did not commensurate with real practice. Gener&shy;ally, nurses showed some responsibility in practicing safe drug administration. Findings in relation to nurses&rsquo; attitude and skills in medication administration were found to be sufficient. However, this finding is parallel with results obtained by Manias et al. (2004) and Shea (1999).</p>\r\n<p>\r\n	Years of experience did not show any influence on the knowledge and practice of nurses except on attitude, which is similar to the study done by Ivey et al. (1996). Findings regarding pharmaco&shy;logical knowledge and practice are re&shy;lated to edu&shy;cational level and are similar to the findings of Boggs et al. (1998).</p>\r\n<p>\r\n	<strong>LIMITATIONS</strong></p>\r\n<p>\r\n	A consensus could not be reached to indicate that knowledge, attitude and practice of nurses is really of an average level in actual practice, even though the findings of this study indicate so, as questions measuring knowledge were simple and lacked depthness. Further&shy;more, it did not cover many aspects of pharmacology. Mathematical skills which is a prerequisite to the performance of many nursing functions such as medica&shy;tion calculation was not tested here. In relation to nurses&rsquo; skill, nurses could have performed well as they were aware of the presence of the researcher.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The findings of this study concluded that nurses in Medical ward, UKMMC were found to have generally an average level of knowledge, practice and attitude in administering oral medication. Despite the fact that it is of adequate standard, it is not sufficient as the nurses are quali&shy;fied nurses working in a teaching hospital and teaching student nurses is part and parcel of nurses&rsquo; role in nursing practice. Nurses need to continually up&shy;date their pharmacological knowledge from time to time in order to be competent in the ad&shy;ministration of drugs.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(427, 424, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Skin is the largest and outermost organ of the human body. The skin is frequently and directly exposed to a pro-oxidative environment, including ultraviolet radia&shy;tion (UV), drugs, and air pollutants. Be&shy;sides external inducers of oxidative at&shy;tack, the skin has to cope with endogen&shy;ous gen&shy;eration of reactive oxygen spe&shy;cies (ROS) and other free radicals, which are con&shy;tinuously produced during physi&shy;ological cellular metabolism.</p>\r\n<p>\r\n	Balance between oxidants and antioxi&shy;dants is needed to minimize molecular, cellular, and tissue damage. However, if the balance is upset in favour of the oxi&shy;dants, oxidative stress could occur and results in oxidative damage. Reactive oxygen species (ROS) are known to cause oxidative modification of DNA, proteins, lipids and small cellular mole&shy;cules (Kang et al. 2005). To counteract the harmful effects of ROS, the various compartments of the skin (epidermis, dermis, subcutis) are equipped with lay&shy;erspecific antioxidant systems to main&shy;tain an equilibrium between ROS and antioxi&shy;dants and thus prevent oxida&shy;tive stress (Thiele and Ekanayake-Mudiyanselage 2007).</p>\r\n<p>\r\n	Skin aging is defined as structural changes of skin as the age increase. Ag&shy;ing is indicated as inability to balance the important functions of skin leading to cell death. The free radical theory of ag&shy;ing explains the importance of antioxi&shy;dants in reducing pigmentation in older people (Chandrosoma &amp; Taylor 2001).</p>\r\n<p>\r\n	The human pigmentary system is de&shy;pendent on the production of the light ab&shy;sorbing biopolymer, melanin, within epi&shy;dermal, ocular and follicular melano&shy;cytes (Nordlund et al. 1998). Melano&shy;cytes within the skin are situated on the basal layer between the dermis and epi&shy;dermis and have a number of dendritic processes that interdigitate with the sur&shy;rounding keratino&shy;cytes. While pigment synthesis occurs within the melanocytes, the majority of pigment within the skin is found in melanin laden vesicles known as melanosomes located within the kerati&shy;nocytes (Sturm et al. 2001).</p>\r\n<p>\r\n	Melanin synthesis is also known as melanogenesis where the pigment mela&shy;nin is formed. The melanin pigments are of no fixed molecular weight but are all derived by enzymatic oxidation of the amino acid tyrosine and eventually pro&shy;duce two types of melanin in mammalian skin which are feomelanin and eumelanin (Prota 1992; Nordlund et al. 1998). Tyro&shy;sinases catalyze two distinct reactions in melanin synthesis: the hydroxylation of tyrosine to 3,4-dihydroxyphenylalanine (DOPA) and the oxidation of DOPA to dopaquinone (Tripathi et al. 1992). The eumelanins are derived from the metabo&shy;lites of DOPAchrome, whereas the pheo-melanins are derived from metabo&shy;lites of 5-S-cysteinylDOPA. The isomeri&shy;zation of DOPAchrome to 5,6-dihydroxy&shy;indole-2-carboxylic acid (DHICA) is cata&shy;lysed by DOPAchrome tautomerase and the oxi&shy;dation of DHICA is performed by a DHICA-oxidase enzyme (Sturm et al. 2001).</p>\r\n<p>\r\n	Important genes involved in melanin synthesis pathway are <em>TYR, TYRP1</em> and <em>TYRP2.</em> Tyrosinase is encoded by the TYR or c-locus that maps to chromo&shy;some 11q14&ndash;21 in humans (Barton et al. 1988) and chromosome 7 in mice, re&shy;spectively. It is composed of five exons and four in&shy;trons (Jimbow et al. 2000). The gene for TYRP1 is 37 kb long and contains eight exons separated by seven introns (Nordlund et al. 1998). The gene for TYRP2 is 60 kb long and contains eight exons and seven introns; all eight exons encode the final protein (Budd and Jackson 1995). Gene and protein struc&shy;tures of tyrosinase, <em>TYRP1 </em>and <em>TYRP2</em> consist of NH<sub>2</sub>- terminal domain of tyrosi&shy;nase which comprises of the NH<sub>2</sub>-terminal signal peptide that is important for intra&shy;cellular trafficking and process&shy;ing, the EGF-like/cysteine-rich domain, two histi&shy;dine-rich regions binding copper with a cysteine-rich region between them (the important catalytic domain), as well as the COOH-terminal hydrophobic trans&shy;membrane segment and cytoplas&shy;mic tail (Kwon 1993). The transmem&shy;brane and cyto&shy;plasmic domains are nec&shy;essary for tar&shy;geting the enzyme to the melanosome (Jimbow et al. 2000; Selaturi 2000)while the NH<sub>2 </sub>terminus cysteine-rich region may serve as a pro&shy;tein binding/regulatory do&shy;main unrelated to enzymatic function.</p>\r\n<p>\r\n	Tocopherols and tocotrienols are sub&shy;families of vitamin E (Brigelius-Flohe &amp; Traber 1999). Both tocopherols and to&shy;cotrienols have isomers, designated as -&alpha;, -&beta;,-&delta; and -&gamma; which differ by the number and position of the methyl groups on the chromanol ring (Machlin 1991). Tocotrie&shy;nol rich fractions are vitamin E that con&shy;sists of 75% alpha-tocotrienol and 25% tocopherol (Mutalib et al. 2003). Toco&shy;trie&shy;nol rich fractions are found in barley, oats, palm, and commercial rice brans (Qureshi &amp; Qureshi 1993).</p>\r\n<p>\r\n	In this study, the anti pigmentation prop&shy;erties of palm tocotrienol rich fraction was evaluated. Primary culture of skin melanocytes was treated with tocotrienol rich fraction to evaluate the effects of tocotrienol rich fraction on melanin syn&shy;thesis by determining melanin content and tyro&shy;sinase enzyme activity. Determi&shy;nation of gene expression involved in the regulation of melanin synthesis such as <em>TYR, TYRP1</em> and <em>TYRP2</em> was also car&shy;ried out.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Cell culture</em></p>\r\n<p>\r\n	Human melanocytes obtained from the fore&shy;skin of nine to 12 year-old boys were grown in Medium 254, which is a basal medium containing essential and non essential amino acids, vitamins, organic com&shy;pounds, trace minerals, and inor&shy;ganic salts (Cascade Biologics, USA). The me&shy;dium was supplemented with Human Melanocyte Growth Supplement-2 (HMGS-2), which contains 0.5% fetal bo&shy;vine serum, 3 ng/ml basic fibroblast growth factor (human recombinant), 0.2% bovine pituitary extract, 3 &micro;g/ml heparin, 0.18 &micro;g/ml hydrocortisone, 5 &micro;g/ml insu&shy;lin, 5 &micro;g/ml transferrin, and 10 ng/ml phorbol 12- myristate 13-acetate. Written consent was obtained from parents or guardians of all subjects.</p>\r\n<p>\r\n	<em>Cell viability assay</em></p>\r\n<p>\r\n	Cell viability was assessed with CellTiter 96* Aqueous Non-Radioactive Cell Pro&shy;liferation Assay (MTS, Promega, USA). The MTS assay em&shy;ploys 3-(4,5-di&shy;me&shy;thyl&shy;thiazol-2-yl)-5-carboxymethoxyphenyl) 2-(4-sulfophenyl)-2H-tetrazolium (MTS) and the electron coupling agent phenazine methosul&shy;phate (PMS). The MTS com&shy;pound is reduced by the dehy&shy;droge&shy;nase en&shy;zymes found in metaboli&shy;cally ac&shy;tive cells into a formazan product that is solu&shy;ble in the medium. The amount of colored formazan product is propor&shy;tional to the number of viable cells. Briefly, 20 &micro;l MTS solution was added to each well and incu&shy;bated in a humidi&shy;fied incubator at 37<sup>0</sup>C in 5% CO<sub>2 </sub>for 2 - 4 hours. The quantity of formazan product present was determined by measuring the absor&shy;b&shy;ance at 490 nm with a micro&shy;titer plate reader (VeraMax Molecular Devices, USA).</p>\r\n<p>\r\n	<em>Determination of melanin content</em></p>\r\n<p>\r\n	Melanin content was determined ac&shy;cording to Huang et al. (2008) with slight modification. The cells (10<sup>5</sup>) were treated with 500&micro;g/ml TRF (Sime Darby Bhd, Malaysia) for 24 hours. Cell pellets were dissolved in 1 ml of 1 N NaOH at 37 &deg;C overnight and centrifuged for 10 minutes at 10,000 x g. The optical density (OD) of the supernatants was measured at 450nm using the &micro;Quant microplate reader. Melanin concentration was calculated by comparing the OD at 450 nm of unknown samples with a standard curve (Figure 2).</p>\r\n<p>\r\n	<em>Determination of cellular tyrosinase activity</em></p>\r\n<p>\r\n	Cellular tyrosinase activity was measured according to Lin et al. (2007), with slight modification. Melanocytes (10<sup>5</sup>) were cultured in 24-well plates for 24 hours followed by 24 hours treatment with 500&micro;g/ml TRF (Sime Darby Bhd, Malay&shy;sia). Cells were then washed and lysed with potassium phosphate-buffered sa&shy;line (PBS) pH 6.8 containing 1% Triton X-100 and ruptured by freezing at -80&deg;C and thawing in a water bath. Cell lysates were then clarified by centrifugation at 10,000 x g for 10 minutes. Protein con&shy;tent was determined using Bradford as&shy;say. Protein concentrations were ad&shy;justed with lysis buffer until each lysate contained the same amount of protein (40 &micro;g). The final reaction mixture in each well contained the cell lysate, 10 &micro;l 2.5 mM L-dopa, and 0.1 M PBS pH 6.8. Absorbance was then measured at 475 nm using the &micro;Quant microplate reader after incubation at 37&deg;C for 1 hour.</p>\r\n<p>\r\n	<em>RNA extraction</em></p>\r\n<p>\r\n	Total RNA from fibroblast cells in differ&shy;ent groups were extracted using TRI Re&shy;agent (Molecular Research Centre, Cin&shy;cinnati OH) according to the manu&shy;fac&shy;turer&rsquo;s instruction. Polyacryl Carrier was added in each extraction to precipi&shy;tate the total RNA. Extracted RNA pellet was then washed with 75% ethanol and dried before being dissolved in RNase and DNase free distilled water (Gibco Invitro&shy;gen Corp.). Total RNA was stored at -80&deg;C immediately after extraction. Yield and purity of the extracted RNA was de&shy;termined by Nanodrop (Thermo Scien&shy;tific).</p>\r\n<p>\r\n	<em>Primer design</em></p>\r\n<p>\r\n	Primers for human GADPH and gene of interest were designed with Primer 3 software and blasted with Genebank da&shy;tabase sequences. The sequence of the primers is shown in Table 1.</p>\r\n<p>\r\n	<em>Real time RT-PCR</em></p>\r\n<p>\r\n	Gene expression of <em>TYR, TYRP1, TYRP2</em> was quantitatively analysed with real time RT-PCR technique. The expression level of each targeted gene was normalized to GAPDH. Primers for hu&shy;man <em>GAPDH, TYR, TYRP1, TYRP2</em> were designed with Primer 3 software and blasted with GeneBank database se&shy;quences in order to obtain primers with high specificity. The efficiency and speci&shy;ficity of each primer set were confirmed with standard curve (Ct value versus se&shy;rial dilution of total RNA) and melting profile evaluation. Real time RT-PCR re&shy;action was performed with 100 ng of total RNA, 400 nM of each primer and iScript One-Step RT-PCR kit with SYBR Green (Biorad) according to the manu&shy;facturer&rsquo;s instruction. Reactions were run using Bio-Rad iCycler with reaction pro&shy;file as follows; cDNA synthesis for 30 min at 50&deg;C; pre-denaturation for 2 min at 94&deg;C; PCR amplification for 38 cycles with 30 sec at 94&deg;C, 30 sec at 60&deg;C and 30 sec at 72&deg;C. This was followed by a melt curve analysis to determine the reaction specificity. Agarose gel electro&shy;phoresis was performed for confirmation of the PCR product. Expression level of each targeted gene was normalized to <em>GAPDH</em>.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	Each experiment was carried out in tripli&shy;cates with at least 3 independent cultures with comparable results. Data are re&shy;ported as mean <u>+</u> SD of at least three experiments. Comparison between groups was made by Student&rsquo;s <em>t</em>-test (two-tailed). P&lt;0.05 was considered sta&shy;tistically significant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Effects of Tocotrienol Rich Fraction on cell viability</em></p>\r\n<p>\r\n	Incubation of melanocytes with different concentrations of tocotrienol rich fraction (100, 200, 300, 400, 500 ug/ml) for 24 hours caused an increase in the number of viable cells (Figure 1). The percentage of the viable cells was increased with in&shy;creased concentration of tocotrienol rich fraction. Tocotrienol rich fraction at 500 ug/ml was used in this study to determine its effects on melanin synthesis.</p>\r\n<p>\r\n	<em>Effects of Tocotrienol Rich Fraction on melanin content and tyrosinase activity. </em></p>\r\n<p>\r\n	Melanin concentration was calculated by comparing the OD at 450 nm of unknown samples with a standard curve (Figure 2). Results showed that treatment with toco&shy;trienol rich fraction caused a signifi&shy;cant reduction in melanin content (Figure 3) (p&lt;0.05) as compared to the untreated control. Tyrosinase activity was signifi- cantly lower (p&lt;0.05) in melanocytes treated with tocotrienol rich fraction (Fig&shy;ure 4) compared to the untreated control.</p>\r\n<p>\r\n	<em>Effects of tocotrienol rich fraction on the expression of TYR, TYRP1 and TYRP2 genes</em></p>\r\n<p>\r\n	The agarose gel electrophoresis showed a single band of PCR product indicating that the primers were specific (Figure 5a). The melting curve analysis showed single and narrow peak as an indication of primer specificity (Figure 5b-e)</p>\r\n<p>\r\n	Results showed that treatment with to&shy;cotrienol rich fraction caused a significant downregulation of <em>TYRP2</em> gene (p&lt;0.05) compared to the untreated control (Fig&shy;ure 6). Expression of <em>TYR</em> and <em>TYRP1</em> genes was not significant from the un&shy;treated control.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Previous study reported that com&shy;pounds with redox properties or (antioxidants) may have anti pigmentation effects by interacting with o-quinones or interacting with copper at the tyrosinase active site, thus avoiding the oxidative polymeriza&shy;tion of melanin intermediates. Redox agents could inhibit second messengers which were able to stimulate epidermal melanogenesis either directly or indirectly by scavenging reactive oxygen species generated in the skin following UV expo&shy;sure (Karg et al. 1993). Our findings showed that melanin content and tyrosi&shy;nase activity were significantly reduced in melanocytes treated with 500 &micro;g/ml to&shy;cotrienol rich fraction.</p>\r\n<p>\r\n	It has been reported that &alpha;-tocopherol derivatives could inhibit tyrosinase <em>in vitro</em> (Shimizu et al. 2001) and inhibit melano&shy;genesis in epidermal melanocytes (Ichihashi et al. 1999). The antioxidant properties of a-tocopherol (25% in to&shy;cotrienol rich fraction), which interfere lipid peroxidation of melanocyte mem&shy;branes and increased intracellular glu&shy;tathione content, could explain its depig&shy;menting effect (Marmol et al. 1993). Other antioxidants such as ascorbic acid interfere with the different steps of melanization by interacting with copper ion at the tyrosinase active site (Gukasyan 2002) or/and reducing dopaquinone as well as blocking DHICA oxidation (Ros et al. 1993) thus causing reduction in pigmentation.</p>\r\n<p>\r\n	Tyrosinase activity was an important and significant parameter to measure melanogenesis in pigment cell culture (Hu 2008). The significant reduction in tyrosinase activity and melanin content in the tocotrienol rich fraction-treated melano-cytes in this study showed inhibi&shy;tion of melanogenesis in these cells. Similar findings were reported in previous studies that used active constituents from formosan apple (Lin et al. 2007), aloesin (Jones et al. 2002), picnogenol (Kim et al. 2008) and anemonin (Huang et al. 2008).</p>\r\n<div>\r\n	<p>\r\n		Melanin synthesis was affected by many factors such as the presence of TRP-1 and TRP-2 proteins, UV expo&shy;sure, prostaglandin, vitamins, growth factors, interleukin, interferon and hor&shy;mones such as alpha-melanocyte stimu&shy;lating hormone, adrenocortropic hormone and endothelin-1 (Fang et al. 2002). Ge&shy;netic control was also involved in melanin synthesis (Rinchik et al. 1993).</p>\r\n	<p>\r\n		Our results showed that <em>TYRP2 </em>geneexpression was significantly reduced in tocotrienol rich fraction-treated melano&shy;cytes while <em>TYR</em> and <em>TYRP1 </em>genesde&shy;creased insignificantly. These findings showed <em>TYRP2 </em>expression was affected by tocotrienol rich fraction treatment but not for <em>TYR</em> and <em>TYRP1</em> genes expres&shy;sion. A previous study reported that anti&shy;oxidants caused reduction in tyrosinase activity but did not affect the tyrosinase related protein itself (Khatib et al. 2005; Kim et al. 2006).</p>\r\n	<p>\r\n		Huang et al. (2008) reported that anemonin, a natural bioactive compound, could regulate tyrosinase-related proteins and mRNA in human melanocytes. Eberle et al. (2001) showed that catechol could inhibit gene expression encoded for TRP-2 protein which was <em>TYRP2</em>.</p>\r\n	<p>\r\n		<em>TYR</em>is controlled by a quantitative rheostat-like switch which allows con&shy;tinuous transcription of <em>TYR </em>meanwhile <em>TYRP1</em> is controlled by binary switch that would allow transcription of genes only at certain time. Therefore <em>TYR</em> has variable control mechanisms such as regulation at transcription level, and post translational mechanism as well as affected by mRNA stability (Hazily et al. 2002).</p>\r\n	<p>\r\n		<em>TYR, TYRP1</em>and <em>TYRP </em>genes have been cloned and extrinsic factors regu&shy;lating their expression were recently identified. It was reported that <em>TYRP1</em> and <em>TYRP2</em> genes may act together to modulate <em>TYR</em> activity (Manga et al. 2000). This may explained the down regulation of <em>TYRP2</em> gene in this study that affect the activity of tyrosinase en&shy;zyme.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		In conclusion, the significant reduction in tyrosinase activity and melanin content in melanocytes treated with tocotrienol rich fraction and down regulation of <em>TYRP2</em> gene confirmed the anti pigmentation property of palm tocotrienol rich fraction, an invaluable finding in the cosmetic in&shy;dustry.</p>\r\n	<p>\r\n		<strong>ACKNOWLEDGEMENT</strong></p>\r\n	<p>\r\n		This study was funded by Sime Darby Bhd, Malaysia grant JJ-001-2008 and Universiti Kebangsaan Malaysia.</p>\r\n</div>\r\n', 1),
(428, 425, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Prolong waiting time in outpatient clin&shy;ics is one of the most frequently en&shy;countered concerns of patients in many of the hos&shy;pitals worldwide and has been a long standing issue that is yet to be solved by health care providers. It is still a hot topic of discussion even today among health care users, their family members and also the health care pro&shy;viders. Patients are increasingly dissatis&shy;fied with the &ldquo;waiting time&rdquo; and &ldquo;duration of consulta&shy;tion&rdquo; at outpatient clinics. Sev&shy;eral studies have ranked waiting time in outpatient clinics as their most major concerns, giving little attention to prob&shy;lems con&shy;cerning health care, staff com&shy;munication, behaviour and attitude (Bursch et al. 1993).&nbsp;</p>\r\n<p>\r\n	The various complaints received from patients are: (1) too much time is spent waiting just to be seen by a doctor, (ii) doctors not punctual and (iii) too short a consultation time. Prolong waiting time at outpatient clinics and a short duration of consultation which has been often deemed as of poor quality has also caused many patients to default treat&shy;ment or seek outpatient treatment in pri&shy;vate hospitals. This has resulted in many patients to voice out their grouses and dissatisfaction through letters of com&shy;plaint to the Director of hos&shy;pitals and to the media. Patients were also observed to become impatient and lose their tem&shy;per, resulting in unpleasant incidences between clinic staff and pa&shy;tients. The patient&rsquo;s rapport with the phy&shy;sician, waiting time before consultation and con&shy;sultation length are contributing factors to the long waiting time that have been identified as possible predictors of patient satisfaction. Therefore, prolong waiting time is the main cause of dissa&shy;tisfaction among outpatients.</p>\r\n<p>\r\n	Prolong waiting time does not only af&shy;fect the patients themselves but also their family members.&nbsp; Being a close-linked family and bounded with strong family ties, majority of the children showers the responsibility of taking care of the health of their aged parents and involve actively in their health care decisions. Many make a point to be around or be present when the parents are being seen by their doc&shy;tors (during consultation time). Therefore, during every hospital visits and follow up appointments, majority of the working children accompany their aged parents to clinic and wait anxiously with their aged parent until they are seen by the doctor. The huge responsibility of sending their aged parents to hospital and then send&shy;ing them back home safely, requires majority of the children to take time off from work, and many had to rush back to work after a long waiting time in the out&shy;patient clinic. In fact, studies have shown that perception of waiting time by pa&shy;tients, parents and family members are often longer than the reality (Harris et al. 2002; Thompson et al. 1996).&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Studies have shown that prolong wait&shy;ing time in outpatient clinics could lead to dissatisfaction among patients. Prolong waiting time can lead to overcrowded clinics and extended waiting time when doctors do not arrive at the clinic at a specific time. Gourdji et al. (2003) has also identified length of time in the wait&shy;ing room as a main element of dissatis&shy;faction among outpatients. Activities like relocating the individual to take blood samples, attending health education class in the clinic itself has been sug&shy;gested to provide a less stressful envi&shy;ronment and reduce patient&rsquo;s distress while waiting.</p>\r\n<p>\r\n	Patient satisfaction with health care services has become one of the most frequently used criteria for evaluating programs or treatment outcomes. Pa&shy;tient&rsquo;s perception of clinic waiting expe&shy;rience was found to affect satisfaction. Patients do underestimate or overesti&shy;mate their actual waiting time. Waiting for less than half an hour from their actual waiting time does not affect their satis&shy;faction but waiting for more than half an hour was found to be unacceptable by patients. Bruce et al. (1998) reported that the patient&rsquo;s perceived waiting time is normally more than the actual time spent waiting. However, patients are satisfied if their waiting time does not exceed more than 40 minutes after arriving on time for an appointment (Huang 1994).</p>\r\n<p>\r\n	Psychological factors could also influ&shy;ence the patient&rsquo;s perceptions of the clinic waiting experience. For instance, unoccupied time feels longer than occu&shy;pied time and uncertain waiting or unex&shy;plained waiting may be seen longer than explained waiting and known finalized waiting. The consequences of long wait&shy;ing can directly impact patient satisfac&shy;tion and health outcomes by generating unnecessary emotional turmoil for the person waiting (Rondeu 1998) and their family members.&nbsp;&nbsp;</p>\r\n<p>\r\n	The Malaysian Ministry of Health (MOH) recognizes the problem of pro&shy;longed waiting times and has suggested ways to improve the waiting time of all outpatient clinics. To solve this problem, the MOH has recommended the stag&shy;gering appointment time which reduces the waiting time to a certain level and encourages hospitals to phase off the traditional system of giving appointments. The Institute of Medicine in America has also recognized the problem of prolong waiting times and has recommended that at least 90% of scheduled patients should be seen within 30 minutes of their scheduled appointment time (target time). Outpatient Clinics Managers have also introduced the patient charter stan&shy;dard guidelines to ensure health care providers minimize waiting time of pa&shy;tients to a satisfactory level.</p>\r\n<p>\r\n	Kurata et al. (1992) in his study has compared patient&rsquo;s and health care pro&shy;viders satisfaction on waiting time and identified 8% of patients and 22% of health care providers were dissatisfied with waiting time. The actual waiting time was estimated to be not more than 45 minutes of their scheduled appointment time. Fernandes et al. (1994) reported that 90% of patients were willing to wait up to one hour from the scheduled ap&shy;pointment time. Another study by Dos Santos et al. (1994), reported that pa&shy;tients had experienced a long waiting time of 188 minutes and 148 minutes (Bangboye &amp; Jarallah 1994).</p>\r\n<p>\r\n	Services at hospitals are expanding, patients seeking health care are in&shy;creasing but improvement in the waiting time and duration of consultation are yet to be achieved. Hospitals have also been awarded with certification of ISO 1999 - 2000, however, the quality of services has not changed and still remains the same or poor and in fact, it has deteri&shy;orated from bad to worse, which would be due to the increasing number of patients. Nurses and doctors play an important role in ensuring that patients are satisfied with the waiting time, duration of consultation and the overall services rendered are of quality. Therefore, identifying patient satisfaction on waiting time and duration of consulta&shy;tion and overall services of the clinic, will enable an organization to continuously improve the quality of services provided.</p>\r\n<p>\r\n	No study has been done locally to ex&shy;amine the waiting time at any of the out&shy;patient clinics in UKMMC. The present study aims to measure the waiting time and duration of consultation and examine pa&shy;tient&rsquo;s level of satisfaction towards wait&shy;ing time and duration of consultation at the Orthopedic Clinic, UKMMC. This study also examined patient&rsquo;s satisfaction towards the overall clinic services.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross-sectional descriptive survey was conducted to assess patient satisfaction on waiting time and duration of consulta&shy;tion among patients attending follow up appointments. A total of 81 patients aged between 18 and 80 years were re&shy;cruited in this study. Patients who came to the clinic without any prior appointment are excluded from the study. Data was collected from June 2004 until April 2005. All patients completed a self-rating structured ques&shy;tionnaire that measured waiting time and duration of consultation. Questionnaires were extracted from the previous studies of Dhanaraj (1997) and Narimah (2004) and were slightly redesigned. Question&shy;naires consisted of five sections: Section A con&shy;sisted of questions on waiting time that was recorded by the observer (re&shy;searcher), Section B consisted of ques&shy;tions on the factors that influence waiting time such as arrival time of the clinic doctor and clinic&rsquo;s filing system (also rec&shy;orded by the researcher), Section C con&shy;sisted of questions that identify patient&rsquo;s expectation of waiting time, Section D evaluated the duration of consultation experienced by the patients, and Section E examined patient satisfaction on both waiting time and duration of consultation. Questionnaires in section E were divided into two sections: Section 1 examined the level of satisfaction of waiting time and duration of consultation whereby Section 2 examined the level of satisfaction to&shy;wards clinic services. Section C, D and E were completed by the patients them&shy;selves. &nbsp;Waiting time referred to the time patient is given the calling number till the time patient is called into the doctor&rsquo;s room. Duration of consultation referred to the time the patient enters and leaves the doctor&rsquo;s room.</p>\r\n<p>\r\n	Waiting time experienced by patients were categorized into two levels:&nbsp; waiting time Level I was the waiting time expe&shy;rienced by patients from the time of re&shy;porting at the registration counter till a waiting number is given. Waiting time Level II was the waiting time experienced by patients from the time the patient re&shy;ceived the waiting number till the patient was called into the doctor&rsquo;s room. Direct observation of waiting time and duration of consultation was also monitored and recorded by the researcher using a timer. These data was compared to the ex&shy;pected time of patients and the actual time reflected on the log charter main&shy;tained in the Orthopedic clinic.</p>\r\n<p>\r\n	A &ldquo;prolonged waiting time&rdquo; was defined as when the patient&rsquo;s actual waiting time which exceeded the total waiting time as reflected in the clinic log which was 67 minutes. The waiting time showed in the clinic log for level I was 5 minutes and level II was 62 minutes.&nbsp; The actual dura&shy;tion of consultation time reflected in the clinic log was 15 to 60 minutes. Duration of consultation time in this study was ca&shy;tegorized into short consultation time (less than 15 minutes), normal consulta&shy;tion time (15 to 60 minutes) and a pro&shy;longed consultation time (more than 60 minutes).</p>\r\n<p>\r\n	The Statistical Package for Social Sci&shy;ences (SPSS) version 12.0 was used to produce descriptive statistics. Data on satisfaction of waiting time was analyzed using a 4-point Likert scale ranging from (1) strongly do not agree to (4) strongly agree. A score of 3 and 4 was deemed as satisfactory and a score of 1 and 2 was deemed as unsatisfactory. Ques&shy;tionnaires designed consisted of positive- and negative-based as to avoid any bias. For positive type of questionnaires, a score of (4) was deemed as strongly agree, (3) as agree, (2) as disagree, and (1) as strongly disagree. As for negative type of questionnaires, a reversed scor&shy;ing method was used: A score of (4) strongly disagree, (3) disagree, (2) agree and (1) as strongly agree. A score of 29 was considered as an average score as total scores were 40. Therefore, total scores of 29 and above were deemed as satisfactory and scores below 29 were deemed as unsatisfactory. A chi square test was used to examine the relationship between satisfactory scores of waiting time, duration of consultation and socio-demographic factors of respondents.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Table 1 highlights the distribution of waiting time experienced by patients in level I. The average waiting time in level I was 16.51 &plusmn;15.99 minutes as compared to the time (5 minutes) reflected in the clinic log. The average expectation of waiting time for level I was 20.46 &plusmn;16.18 minutes. Majority (34.6%) expected a waiting time from 10-19 minutes in level I. Only seven (8.6%) patients expected a waiting time of 50 minutes and above. Majority (45.7%) of the patients waited less than 10 minutes, which was lower than their expected time but was in line with the targeted time reflected in the clinic log. The waiting time range for level I was 1 &ndash; 97 minutes, which is higher than the expected waiting time of 1 &ndash; 75 minutes. In comparison with the actual waiting time in which majority (45.7%) waited less than 10 minutes, 23.5% of patients expected such a waiting time. Analysis also indicated that only one pa&shy;tient experienced the actual waiting time of 1 minute.</p>\r\n<p>\r\n	Table 2 highlights the distribution of waiting time experienced by patients in level II. The average waiting time in level II was 127.15 &plusmn; 48.65 minutes as com&shy;pared to the time (62 minutes) reflected in the clinic log. The average expected time was 44.63 &plusmn; 30.33 minutes. Twenty-eight (34.5%) of the patients experienced a waiting time of 150 minutes and above. The waiting time range for level II was 44 &ndash; 267 minutes. In comparison with the actual waiting time, majority (34.6%)of patients expected a waiting time of 30 &ndash; 59 minutes, but in actual waiting time, only 5 patients (6.2%) experienced such a waiting time. Only one patient (1.2%) expected a waiting time of 150 minutes and above but in actual waiting time, majority of the patients, 28 (34.5%) expe&shy;rienced such a long waiting time. It goes to indicate that the actual waiting time was significantly higher than the ex&shy;pected time.</p>\r\n<p>\r\n	Table 3 highlights the distribution of consultation time experienced by pa&shy;tients. The average duration of consulta&shy;tion experienced by respondents were 12.79 &plusmn;6.85 minutes as compared to the time range (15 to 60 minutes) reflected in the clinic log. The length of time spent on majority (65.4%) of patients was less than 15 minutes. Only 3 (3.7%) patients experienced a lengthy consultation time between 30 &ndash; 44 minutes compared to the 25 (30.9%) that expected such a time. Majority (40.7%) expected a long duration of consultation of 60 minutes and above, and the duration of consulta&shy;tion of less than 15 minutes is considered as short consultation. Findings indicate a significant difference in the duration of consultation between the expected and the actual duration of consultation (p&lt;0.05).</p>\r\n<p>\r\n	The average total waiting time expe&shy;ri&shy;enced by respondents before doctor&rsquo;s consultation was 143.65 &plusmn; 53.54 minutes as compared to the time (67 minutes) reflected in the clinic log (Table 4). A number of 29 patients had a waiting time of 100 &ndash; 149 minutes and the other 29 (35.8%) patients had a waiting time of 150 &ndash; 199 minutes. The minimum and maximum waiting time was 46 minutes and 300 minutes, respectively. Findings indicate a significant difference between the actual and the expected total waiting time of the patients (p&lt;0.001) and more than twice the standard time reflected in the clinic log.</p>\r\n<p>\r\n	Overall satisfaction towards total wait&shy;ing time and duration of consultation was relatively low, 29.6% (n=24) and 42.0% (n=34), respectively (Table 5). Average total waiting time was significantly longer in the dissatisfied group (p=0.014) and the duration of consultation was signifi&shy;cantly shorter in the dissatisfied group as compared to the satisfied group. Further analysis indicated that there was no sig&shy;nificant association between socio-de&shy;mographic factors and patient&rsquo;s overall satisfaction (Table 6).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Patient satisfaction has been used as a significant indicator to measure the qual&shy;ity of health care (Rondeau 1998). It has become increasingly important in today&rsquo;s healthcare. Therefore, this study was un&shy;dertaken to identify the waiting time and duration of consultation and patient sa&shy;tisfaction of waiting time and overall clinic services at the Orthopedic Clinic, UKMMC.</p>\r\n<p>\r\n	<em>Long Waiting Time</em></p>\r\n<p>\r\n	The issue of waiting too long to be seen by a doctor has been a long-standing issue not only in all outpatient clinics in Malaysia but also in other countries. Several studies by Jackovitz (1999), Luther (1996), Mc Kinnon et al. (1998), Thomas &amp; Frank (1997) and Rondeu (1998) have indicated that long waiting time in outpatient clinics had caused patient dissatisfaction of hospital services and indirectly affected the quality of health care. Findings of the above stu-dies are similar to the findings of this study as majority of patients in outpatient clinics experienced a long waiting time. Although the time duration to be given the waiting number was acceptable and fulfilled the expectation of the patients, the time duration from the time the patient received the waiting number until the pa&not;tient was called into the doctor&rsquo;s room was way too far from the patients&rsquo; expectation. The waiting time had also exceeded the standard waiting time that should be achieved by any government hospitals in which waiting time for patients should not be more than two hours or 120 minutes (Khalis 2005). This could probably be due to the clinic session that started later than the given appointment and the current appointment system (clustered appointment system) that was not really effective. However, hospitals in other countries have managed to rectify the problems of long waiting time by implementing the staggering time in order to provide quality health care.</p>\r\n<p>\r\n	Based on observations, majority of doctors came to start their clinic at 9:30 am. The earliest doctor present was rec-orded at 8:45 am and the latest was by 11:00 am. Doctors&rsquo; delay in attending their clinics was due to the tasks that they have to complete in the Orthopedic wards. All patients in the clinic were given clustered appointments, from 8:00 am to 11:00 am. This caused the patients to wait longer as registration at the counter only starts at 8:00 am.<br />\r\n	<br />\r\n	<em> Short Consultation Time</em><br />\r\n	<br />\r\n	The findings of this study also showed that the duration of consultation did not fulfill the expectation of patients. The du-ration of consultation stated in the clinic charter, which was supposed to be between 15 to 60 mi&not;nutes was not achieved. The short duration could probably be due to follow-up appoint-ments, which do not require thorough and long evaluation of the patients as compared to the new cases. It was ob-served that there was a lack of communi-cation or discussion regarding the pa-tient&rsquo;s disease between the doctor and the patient. This could be due to the doctor who wanted to expedite the con-sultation process due to the large number of patients or to ensure that the clinic operation ended before lunch time.<br />\r\n	<br />\r\n	<em> Patients Satisfaction</em><br />\r\n	<br />\r\n	The findings showed that the level of pa-tients satisfaction on waiting time and consultation time were low. A total of 70.4% and 58.0% of the respondents were dissatisfied with the waiting time and the consultation time, respectively. This is in accordance with an earlier study done by Mowen et al. (1993) in which he reported that patients who waited longer than the expected time had a lower satisfaction level as compared to the patients in which their expected waiting time was fulfilled.</p>\r\n<p>\r\n	Many previous studies such as by Put-nam et al. (1985), Linn and Sheldon (1982) and Hall and Dornan (1990) showed a positive relationship between satisfaction level and age. Hall and Dornan (1990) found that aged patients are less demanding and more satisfied with the services received. Meanwhile, Bar-Dayan et al. (2002) stated that young patients had lower satisfaction level as compared to the older ones. A similar trend could also be seen in this study. This could be due to frequent visits to the clinic and long involvement of patients, especially those with chronic diseases that they began to develop good rapport with the health services provider until the patients assumed that the services are channeled according to their needs. Ac&not;cording to Aljunid (1995), patients who came frequently to the clinic would have a change of perception or adaptation and indirectly would follow the requirements of hospital services.</p>\r\n<p>\r\n	Fox and Stroms (1981) who conducted a study in several hospitals in Baltimore, United States, reported that lower income group showed higher satisfaction level with the services provided as compared to the higher income group. Alpert et al. (1970) found that people with lower in&not;come levels were more satisfied with the services provided. However, there was no significant association between customers&rsquo; satisfaction and their income (Pascoe &amp;&nbsp; Atkinson 1983; Larsen et al. 1997; Korsh et al. 1968). Dhanaraj (1997) found out that women, lower-educated people, elderly, pensioners and lower-income people were more satisfied with the services as compared to their counterparts. Some pattern of associations could be seen in this study where younger age, higher education level, working and higher income group were associated with low satisfaction level. However, all of these associations were not significant.<br />\r\n	<br />\r\n	<em> Overall Satisfaction on Clinic Services</em><br />\r\n	<br />\r\n	In general, respondents showed a mod-erate satisfaction level on the services provided by the Orthopedic Clinic, UKMMC. The mean score of satisfac&not;tion was comparable to the standard score of 29, and a total of 56.8% of the respon-dents satisfied with the clinic services.<br />\r\n	A study conducted in a Mother and Child Health Clinic in Muar, Johor (Mukhtar et al. 1995) showed a satisfaction level of 70% whereas another study done by Narimah Yusof (2004) on antenatal services in the Larut Matang District Health Clinic showed the satisfaction level to be 87.1%.&nbsp; Dhanaraj (1997) also conducted a study at the outpatient unit, Sabak Bernam Hospital, reported the satisfaction level to be 36.9%. However, there were no studies done in relation to waiting time at Orthopedic Clinics. A com&not;parison cannot be made here as they are just ordinary clinics and does not fall under the category of a teaching hospital.</p>\r\n<p>\r\n	The overall satisfaction score by eth-nicity were comparable to the standard score (29) where the Malays showed a score of 28.72 and the non-Malays showed a score of 29.82. No significant difference was noted between the two. According to Weiss (1988), the associa-tion between ethnicity and patients&rsquo; sa-tisfaction on clinic services are inconsistent, and Krol &amp; Nord Lund (1983) also reported that many studies did not show any association between ethnicity and patient&rsquo;s satisfaction.</p>\r\n<p>\r\n	To improve patient satisfaction in outpatient clinics, health care workers should be sensitive to patient&rsquo;s grouses on prolong waiting time. Steps should be taken to reduce the waiting time. Nurses who manage the clinic have to ensure that patient&rsquo;s need not wait longer to see the doctor. Doctor&rsquo;s are to ensure that patient&rsquo;s are not unnecessarily made to wait during the consultation period.</p>\r\n<p>\r\n	Prolong waiting time in outpatient clin-ics is not only an issue in Malaysian hospitals but worldwide. Other countries also face the same problem of prolong waiting time. Studies have been undertaken in outpatient clinics by many researchers and results have helped many hospitals to improve the overall outpatient clinic services tremendously by adopting the staggering appointment time.&nbsp; Not many outpatient clinics in the local hospitals have adopted the staggering system of appointment recommended by the Ministry of Health of Malaysia for some un&not;known reasons<br />\r\n	<br />\r\n	<strong>CONCLUSION</strong><br />\r\n	<br />\r\n	Patients are dissatisfied with the waiting time in the Orthopedic Clinic as it is not in accordance with the waiting time reflected in the clinic log. This study pro-vided important information which can be helpful for nurse managers, doctors and clinic staff of Orthopedics Clinics to fur-ther reduce waiting time and duration of consultation and at the same time im-prove the satisfaction level of patients in the Orthopedic Clinic. Waiting time and duration of consultation are important components of patient satisfaction.<br />\r\n	It is strongly recommended here that the staggering system of appointment recommended by the Ministry of Health, Malaysia, be adopted to reduce pa&not;tient waiting time. Some outpatient clinics in UKMMC and other private hospitals have adopted the staggering system of ap-pointment and have successfully reduced the waiting time.</p>\r\n<p>\r\n	Conducting morning ward rounds be-fore attending the Outpatient Clinic has been identified as one of the main contributing factors in prolong waiting time. It is recommended that to reduce patient waiting time, only a few doctors should be as&not;signed to do the morning ward round. Doctors who are not involved in the morning ward round are required to start seeing patients at the clinic at 8am. On the other hand, doctors who come down to the clinic after their morning ward round should be allowed to see patients whose appointment starts from 9.30am onwards. This recommended practice will not only reduce patients&rsquo; waiting time, but also allow doctors to finish their clinic before lunch time and allow a good rest during lunch time.&nbsp; Doctors, Nurse Manager in charge of the clinic and clinic nurses should communicate effectively with each other and work hand in hand to reduce prolong waiting time of patients at the Orthopedic Clinic.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(429, 426, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Post operative pain is an expected ad&shy;verse outcome following surgery and it often delays mobilization and overall re&shy;covery (Bodian et al. 2001; Clarke et al. 1996). Moderate to severe post operative pain is unpleasant but treatable. Ade&shy;quate level of knowledge and positive attitude are essential components in the delivery of post operative pain manage&shy;ment (De Rond et al. 2000; Bowman, 1994). Ineffective pain management con&shy;tinues to be a complaint of hospita&shy;lized patients despite the emphasis laid on the patient&rsquo;s right to appropriate pain man&shy;agement and the increased aware&shy;ness of the detrimental effects of pain that is in&shy;adequately treated and managed (Idvall et al. 2002; Howell et al. 2000).</p>\r\n<p>\r\n	Pain is defined by the American Pain Society Quality of Care Committee, as an unpleasant sensory and emotional expe&shy;rience associated with actual or potential tissue damage, or described by the pa&shy;tients in terms of such damage (Lin, 2001). According to McCaffery et al. (2002) the most accurate definition of pain is &ldquo;Whatever the experiencing per&shy;son says it is, existing whenever he says it does&rdquo;. This definition of pain has impli&shy;cations for registered nurses as they as&shy;sess pain. The patients experiencing pain only can describe the characteristic of their pain.</p>\r\n<p>\r\n	Insufficient education and training for nurses and patients were amongst the issues reported as poor post operative pain management (Dalton et al. 2001; De Rond et al. 1999). Although studies have shown that pain education pro&shy;grams in&shy;crease nurses&rsquo; knowledge and improve attitudes towards pain manage&shy;ment, the management of post operative pain by nurses still remains a problem (Goodrich 2006; Elliot et al. 1996). Many nurses, are still relying on their personal opinion about patient&rsquo;s pain, rather than using their recorded assess&shy;ment to assist them to choose appropri&shy;ate opioid doses. Ap&shy;propriate pain as&shy;sessment tools are not utilized on a reg&shy;ular basis in acute care settings and this also contributes to un&shy;der-treatment of pain (McCaffery et al. 2000; Clarke et al. 1996).</p>\r\n<p>\r\n	Pain management has been an integral part of nursing practice for many years, yet there are still too many who lack the ba&shy;sic knowledge necessary to manage pain appropriately (McCaffery et al. 2000; Puntilo et al. 1997). Further, unrelieved post operative pain may be harmful and adversely affect the quality of life amongst post operative patient (Howell et al. 2000; Watt-Watson et al. 2000). This study was designed, therefore to deter&shy;mine nurses&rsquo; knowledge and attitude to&shy;wards post operative pain management at UKMMC. The data collected provides timely, and baseline information related to nurses&rsquo; knowledge and attitudes to&shy;wards post operative pain management at UKMMC. These data may be used as a foundation for comparisons in future research on pain management.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A cross sectional study was conducted in three surgical wards and the Intensive Care Unit (ICU) of UKMMC from Febru&shy;ary to April 2008. A questionnaire of 28 items comprising two domains: know&shy;ledge and attitude towards post operative pain management was administered. It was adopted and modified from Mc Caffery et al. (1995). It was an objective assessment tool to measure the nurses&rsquo; knowledge and attitude with regard to pain control. The first 18 questions were of the &ldquo;true or false&rdquo; type related to nurses&rsquo; knowledge towards post opera&shy;tive pain management. The last ten questions delved into their attitude to&shy;wards pain management. A Likert&rsquo;s scale ranging from 1 to 5 (1 indicating &ldquo;strongly disagree&rdquo; and 5 &ldquo;strongly agree&rdquo;) was used. Questionnaires were translated to Bahasa Malaysia and translated back again. Ninety six sets of questionnaires were given to nurses in surgical ward one, two and three and in&shy;tensive care unit (ICU) of UKMMC. There were 84 respondents who successfully responded to this study. Ethics approval was ob&shy;tained from Universiti Kebang&shy;saan Ma&shy;laysia Ethic Committee (FF-040-2008) and permission was granted by the UKMMC director and nursing services.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The response rate of this study was 84 respondents (88%) completed the ques&shy;tionnaire while 12 respondents (12%) did not, despite being given the question&shy;naire. The socio-demographic data of respondents were as shown in Table 1.</p>\r\n<div>\r\n	<p>\r\n		Results showed that 58 respondents (69%) reported to possess moderate level of knowledge towards post opera&shy;tive pain management; 21 respondents (25%) scored high level of knowledge and 5 respondents (6%) had low score of knowledge towards post operative pain management. Table 2, showed the rates of correct answers to questions related to knowledge in post operative manage&shy;ment.&nbsp;</p>\r\n	<p>\r\n		<em>Nurses&rsquo; attitude towards post operative pain management</em></p>\r\n	<p>\r\n		Positive attitude towards post operative pain management was reported by 66 respondents (79%) while 18 respondents (21%) had poor or negative attitude. The respondents&rsquo; attitude towards post oper&shy;ative pain management questions cor&shy;rectly answered is shown in Table 3.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		This study, which measured nurse&rsquo;s knowledge and attitude regarding post operative pain management, suggested that a knowledge deficit does exist. This is consistent with previous research findings (Idvall et al. 2002; De Rond et al. 2000; Coyne et al. 1999). In this study, 58 respondents (69%) scored moderate level of knowledge towards post opera&shy;tive pain management. This is higher than that reported by Cason et al. (1999) in which the same questions were ans&shy;wered correctly by 63.64% of the res&shy;pondents. Cason et al. (1999) also found that the nurses in their sample answered general questions about medications for pain better than specific pharmacologic questions about pain management. The findings in this study were in accordance with earlier findings by Watt-Watson et al. (2000), which showed that many nurses had an inaccurate know&shy;ledge base about common pharmacologic&shy; agents used in pain control. This inadequate knowledge base accounted for the possibility of un&shy;der-treatment of patients&rsquo; post operative pain.</p>\r\n	<p>\r\n		The contribution of inadequate know&shy;ledge and nurse&rsquo;s negative attitudes to&shy;wards the pain experience has been&shy; implicated in the nurses&rsquo; inadequate man&shy;agement of patients in pain (Dalton et al. 2001; De Rond et al. 1999; Bownan, 1994). The findings of this study reported that pharmacologic interven&shy;tions are a major area of concern. 69 respondents (81%) in the study acknowl&shy;edged that patients might think that pain and suffer&shy;ings are necessary, based on one&rsquo;s reli&shy;gious belief. This question dem&shy;onstrated the perceived importance of religious up&shy;bringing and how this may impact upon one&rsquo;s personal opinion and feelings about pain and suffering.&nbsp;</p>\r\n	<p>\r\n		In the Malaysian scenario, aggravating cultural values and beliefs affect many individuals who believed that pain is a challenge bestowed by God, thus one tends to tolerate the pain rather than re&shy;porting the intensity of pain to healthcare staff.&nbsp; Inability to tolerate pain causes one to loss face and self esteem and society may consider this person a coward.&nbsp; Nurses in this present study may expect patients to bear a certain amount of pain and suffering.&nbsp; Clarke et al. (1996) noted in their study that very few nurses felt strongly that patients can and should be maintained in a pain-free state.&nbsp; Majority of nurses would elect to reduce pain ra&shy;ther than to relieve it entirely. The find&shy;ings from earlier studies are similar to those of the current study. Another possible explanation is the misconception involving the use of opioids. Brunier et al. (1995) found that 30% of the nurses sur&shy;veyed falsely believed that 25% of the patients with pain will be addicted to opioids. Nurses may feel that some pa&shy;tients are pain medication seeking as a result of addiction.</p>\r\n	<p>\r\n		There were significant differences found in nurses&rsquo; academic qualifications and attitude towards post operative pain management. Results of this study are congruent with previous studies, which supported the idea that those with higher academic qualifications had better at-&shy;<br clear="all" />\r\n		titude scores (Goodrich 2006; McCaffery et al. 2000; Clarke et al. 1996). Several authors have reported that although nurses have become better informed over the years, there are many who still lack the basic knowledge on appropriate pain management (Watt-Watson et al. 2000; McCaffery et al. 1999; Punntillo et al. 1997).</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		We conclude that nurses in UKMMC possess moderate knowledge and posi&shy;tive attitude towards post operative man&shy;agement. Continuous education in pain management is crucial to improve nurses&rsquo; knowledge and attitude towards post operative management. The results of this study provided a framework for the development and im&shy;plementation of con&shy;tinuing education programs for nursing staff which can en&shy;hance the quality of patient care in post operative pain man&shy;agement. Therefore, it is imperative that pain assessment should be included as the fifth cardinal vital signs in the nursing curriculum.</p>\r\n	<p>\r\n		<strong>ACKNOWLEDGEMENT</strong></p>\r\n	<p>\r\n		Special thanks to Prof. Dr. Baharudin Hj. Omar and Associate Professor Dr. Zaleha Md Isa for their contributions in editing and interpreting the results. In addition, thanks to Prof. Dr. Rohaizak Muhamad (Deputy Dean of Research Unit), and Puan Hamidah Hassan (Head of Department of Nursing) for their sup&shy;port in this study.&nbsp; Last but not least, to all nurses who participated in this study.</p>\r\n</div>\r\n', 1),
(430, 427, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Long waiting time for elective operations has been used as a key performance index of inefficiency of hospital services (Buhaug, 2002). Waiting for treatment may have advance consequences on the natural history of the disease. The longer the wait, the higher the possibility of health impairment, the lower the prob&shy;ability of full recovery (Jofre-Bonet, 2000). These include worsening symp&shy;toms and deteriorating quality of life re&shy;sulting in substantial limitation of activi&shy;ties (Derret et al. 1999). The chances of returning to work after surgery may strongly be influenced by the length of time on the waiting list (Rossvoll et al. 1993).</p>\r\n<p>\r\n	Factors related to waiting times vary depending on hospitals and individual surgeons within hospitals (Cregan, 2005). Hospital services can either be a tax financed public treatment provider with a fixed quality or a private provider with a range of different qualities. Con&shy;sumer&rsquo;s covariates such as income, socio-demographic characteristics and health status, and the quality of treatment provided by the public system may influ&shy;ence the decision on timing of surgery (Jofre-Bonet, 2000). The reasons for elective surgery cancellations (Cregan 2005) include lack of theatre time, lack of postoperative beds, cancellation by pa-tient or carer, patient clinic change and procedural reasons.</p>\r\n<p>\r\n	The decision to be on a waiting list is determined by individual surgeons, using no common criteria (Derret et al. 1999).&nbsp; In general, the waiting time from ortho&shy;pedic consultation to surgery appears unrelated to the level of urgency (Conner-Spady et al. 2005). There is no reliable means of assessing the relative priority of patients on waiting lists. It is also impossible to manage waiting lists rationally to ensure that patients with the highest urgency are served first (Noseworthy et al. 2002).</p>\r\n<p>\r\n	Studies related to waiting times for elective surgery varied in terms of patient setting, main objectives and methodol&shy;ogy. This study tries to assess the wait&shy;ing times for elective surgery in a teach&shy;ing hospital and to identify factors influ&shy;encing the waiting times. These in&shy;clude patient factors (age, gender, occu&shy;pational status and ethnicity); disease factors (se&shy;verity and co-morbidity) and temporal factors affecting services. We hope to give recommendations on man&shy;aging waiting times for elective surgery in teaching hospitals, based on the findings from this study.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Across-sectional study of waiting times to surgery was undertaken by using data<br clear="all" />\r\n	obtained from patient&rsquo;s records coded according to case-mix system based on the International Refined Diagnosis Re&shy;lated Group (IR-DRG). Sample size of two proportions (independent observa&shy;tions), required a total of 366 records (183 per group). Due to limitation of case notes coded according to case-mix, only 267 records (70% power) of elective cases in a teaching hospital are collected for analysis. The study was approved by the Research and Ethics Committee of the hospital.</p>\r\n<p>\r\n	Two dependent variables: T1 and T2 are measured. T1 represents waiting time from the time patient was first seen at the specialist clinic until surgery is done. Waiting times are converted into binary data based on the median waiting times of less than and 6 months or more as short waits and long waits respectively (Mahon et al. 2002; Lofvendahl et al. 2005). T2 represents waiting time from the decision date for surgery to the actual surgery. Based on the studies done by DeCoster et al (1999) and Conner-Spady et al (2005), waiting times are also con&shy;verted into binary data using the median waiting time of less than and one month or more as short waits and long waits respectively. The independent variables are patient factors (age, gender, occupa&shy;tional status and ethnicity); disease fac&shy;tors (severity and co-morbidity) and tem&shy;poral factors affecting services.</p>\r\n<p>\r\n	Hospital records information included date placed in the operation book, total cost for the treatment and surgery, and total cost borne by the patient. Data was analysed using Statistical Package for Social Sciences (SPSS) version 11.0. The Chi-square test was used for asso&shy;ciations between categorical variables and the Mann-Whitney U test to deter&shy;mine whether there is any significant dif&shy;ference between total cost for treatment and surgery, and total cost borne by the patient with T1 and T2 waiting time. Lo&shy;gistic regression was performed to see any association be&shy;tween both waiting time and the inde&shy;pendent variables.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	This study showed that the median wait&shy;ing times for T1 and T2 were 23 weeks (5.75 months) and 5.86 weeks (1.47 months) respectively (Table 1).</p>\r\n<p>\r\n	With regards to the temporal factors, there is no obvious trend seen in the dis&shy;tribution of cases between January 2003 till June 2004 (Figure 1). The mean hos&shy;pital stay is 7.91&plusmn;4.85 days.</p>\r\n<p>\r\n	The significant factors associated with T1 waiting time were age and co-morbidity but only ethnicity was significantly asso&shy;ciated with T2 (Table 2 and Table 3). Ta&shy;ble 4 and 5 showed results of the Mann-Whitney U-test of waiting times with the total cost of the treatment and cost borne by patients.&nbsp; The median total cost of treatment was RM815.00 (interquartile range of RM1486.75) and the median cost borne by patients was RM40.50 (in&shy;terquartile range of RM474.00).</p>\r\n<p>\r\n	There was no significant difference between waiting time T1 and cost borne by patient (p=0.12), and with total cost of treatment (p=0.08). There was also no significant difference between the waiting time T2 and cost borne by patients (p=0.93), and also with the total cost of treatment (p=0.25).</p>\r\n<p>\r\n	However, multiple logistic regression showed that patients who were paid by a third party, (OR) of 1.97 (95% CI:1.05-3.72) were significantly more likely to wait longer than those who paid out of pocket for the surgery (Table 6). Nevertheless, for T2, women, (OR) of 2.29 (95% CI: 1.19-4.42), Indian, (OR) of 2.50 (95%CI: 1.16-5.38) and those who are self-em&shy;ployed (OR) of 4.28 (95%CI: 1.23-14.97) were all significantly more likely to have shorter waiting time (Table 7).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	In the present study, the median waiting time for T1 and T2 were 23 weeks (5.75 months) and 5.86 weeks (1.47 months) respectively. The finding for T1 was similar to the study done by Conner-Spady et al. (2005) in Canada with the median waiting time of 21.07 weeks and by Fielden et al. (2005) in New Zealand with a median of 5.1 months for arthro&shy;plasty. However, there are not many studies done to determine the waiting time for T2, because there is no field in the administrative data to indicate when each patient and surgeon made the deci&shy;sion to proceed with surgery, and hence, it is difficult to get the time when the date of surgery was given to the pa&shy;tient (DeCoster et al. 1999; Derret et al. 1999). The median waiting time for hip replace&shy;ment was 5.6 months, 2.1 months for spine surgery and 1.8 months for athro&shy;scopic knee surgery (Lofvendahl et al. 2005). The discrepancies seen here are due to other complexities in&nbsp; determining the &lsquo;trigger point&rsquo; to the waiting list.</p>\r\n<p>\r\n	Age factor and ethnicity have largely been neglected by previous studies. In this series, age factor and patient co-morbidity significantly influence the wait&shy;ing time T1 and ethnicity has significantly attribute to T2. This was supported by DeCoster et al (1999) whereby, the wait&shy;ing time for older patients were shorter because they were likely to be retired and readily available on short notice for surgery. Age was a significant predictor of maximum acceptable waiting time with older patients preferring shorter maximum acceptable waiting time (Conner-Spady et al. 2005). As for ethnicity, ac&shy;cording to Hacker et al (2004), there were no stud&shy;ies examining ethnicity and waiting times. According to Shwartz et al (1996), the presence of co-morbidities was an im&shy;portant determinant of costs because it increase the total burden.</p>\r\n<p>\r\n	Based on multiple logistic regression for T1, patients who were paid by a third party were significantly more likely to wait longer than those who paid out of their own pocket. This may be due to proc&shy;essing delay in application for claims re&shy;lated to admission and surgery; restric&shy;tion due to limited coverage by the policy or patient&rsquo;s coverage is already at the maximum limit. Dowling (1997) noted that fund-holding patients in United King&shy;dom have shorter waiting times for sur&shy;gery than non-fund holding patients. This may be because fund-holding practices are funded over generously.&nbsp;</p>\r\n<p>\r\n	For T2, women, Indian patients and those who are self-employed were likely to have a shorter waiting time. Females were less likely to be referred and fre&shy;quently need urgent surgery (Hacker et al. 2004). Working patients had signifi&shy;cantly shorter waiting times than non-working patients (Lofvendahl et al. 2005). This is due to the fact that by receiving surgical treatment more quickly, they would be subject to fewer interruptions at work (DeCoster et al. 1999).</p>\r\n<p>\r\n	This study is not without limitations.&nbsp; Administrative data always lack clinical information. The study&rsquo;s generalization is also questionable as this study was done in a teaching hospital with a case-mix system in place. Finally, the interpretation of the results is limited by the absence of information on other variables that might have an impact on the waiting times.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The results indicate that the predictors of waiting time are age, comorbidity, ethnic&shy;ity, occupation and the method of paying for services rendered by the pa&shy;tients. Another important factor for time spent on waiting list was hospital type, whereby a possible explanation for longer waiting time in a teaching hospital could be that elective surgery is frequently delayed due to emergency cases (Lofvendahl et al. 2005).&nbsp;</p>\r\n<p>\r\n	In non-life-threatening conditions, in which there is insufficient evidence on the long-term effects of waiting times on clinical outcomes, patient, physician and public input to decision making is critical to a fair process for establishing stan&shy;dards for acceptable waiting times for scheduled procedures. Attention should be focused on the role of hospital-related factors: scheduling admissions, evalua&shy;tion of priority strategies, allocation of operating theatres, capacity planning for intensive care units, bed capacity analy&shy;sis, planning, blood bank management, and the use of quality of life instruments in the future studies.&nbsp; Future research should also include study of maximum acceptable waiting times (Conner-Spady et al.2005) as it relates to ideal waiting time, patient information, perceived eq&shy;uity, past experience with waiting, and patient satisfaction. For an acceptable service, a better method in prioritizing length of waiting time according to symptomatic experience is required.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	The author would like to thank En. Rosman from case-mix Unit and Dr Feisul Idzwan for their assistance and co-operation.</p>\r\n', 1),
(431, 428, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	<em>Staphylococcus epidermidis</em>(coagulase negative staphylococcus) is emerging as an increasingly important pathogen in the immunocompromised host. <em>S. epidermi&shy;dis</em> has been isolated as an etiological agent in critical and life-threatening con&shy;ditions such as septicemia in preterm infants (von Eiff et al. 2001). We report here a case of necrotizing pneumonia, rarely attributed to <em>S.&nbsp; epidermidis</em> in a term infant who had earlier undergone surgical repair of tracheo-esophageal fistula.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A three-day old term male infant under&shy;went end-to-end anastomosis and fistu&shy;lectomy for esophageal atresia and tra&shy;cheo-esophageal fistula.&nbsp; Two days post-operatively, he was extubated, but devel&shy;oped stridor and significant respiratory distress requiring re-ventilation. Several attempts at extubation failed; hence a bronchoscopic examination was per&shy;formed at day 10, which revealed signifi&shy;cant edema at the fistulectomy site (level of the third thoracic vertebra). He was commenced on a tapering course of dex&shy;amethasone therapy over five days to facilitate extubation. However, on day 15 of life, he had evidence of sepsis and increased oxygen and ventilatory re&shy;quirements. He was febrile (tempera&shy;ture of 38<sup>o </sup>Celsius) and there were coarse crepitations on auscultation of both lung fields. A full blood count showed low total white cell number (1.4 X 10<sup>9</sup> /L) with neutropenia (0.9 X 10<sup>9</sup> /L) and thrombo&shy;cytopenia (93 X 10<sup>9</sup> /L). Se&shy;rum C-reactive protein (CRP) was also elevated (21 mg/dL). A chest radiograph showed ha&shy;ziness of the right lung and multiple rounded radiolucent foci with no air-fluid levels (Figure 1). Ultrasono&shy;graphic ex&shy;amination of the lungs con&shy;firmed areas of consolidation but there was no evidence of abscess or empyema formation. A ga&shy;strograffin con&shy;trast study did not reveal evidence of an anasto&shy;motic leak. Cul&shy;tures of blood, tra&shy;cheal aspirates and the tip of the extra-pleural chest tube inserted intra-opera&shy;tively grew coagulase nega&shy;tive <em>S. epi&shy;dermidis</em>. A diagnosis of <em>S. epi&shy;dermidis</em> necrotizing pneumonia with septicemia was made and he was treated with intra&shy;venous tei&shy;coplanin and genta&shy;micin for four weeks followed by two weeks of erythromycin. No percutaneous aspira&shy;tion or pleural drainage was re&shy;quired. A repeat chest radiograph after the first three weeks of treatment showed pneumatocele formation with some clearance of the pneumonia (Figure 2). The in&shy;fant was success&shy;fully extubated from mechanical ventila&shy;tion to a brief pe&shy;riod of continuous posi&shy;tive air-way pres&shy;sure support fol&shy;lowed by nasal oxygen sup&shy;plementation. The course of his hos&shy;pital stay was not com&shy;plicated by pneu&shy;motho&shy;rax or bron&shy;cho&shy;pleural fistula. The infant was dis&shy;charged well after six weeks of antimi&shy;crobial ther&shy;apy. Complete radio&shy;graphic resolution of the pneumoto&shy;celes was seen on follow-up six months later.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Necrotizing pneumonia or pulmonary gangrene is a severe complication of bacterial pneumonia. More commonly reported in adults, this condition is rare in children and even more so among neo&shy;nates. Organisms causing this cavitating form of pneumonia include <em>Streptococcus pneumoniae</em>, <em>Streptococcus pyogenes, Mycoplasma pneumoniae</em>(Hacimusta&shy;faoglu et al. 2004), <em>Pseudomonas aeru&shy;ginosa </em>(Reich 1993) and methicillin-re&shy;sistant <em>Staphyloccus aureus </em>(McAdams et al. 2005). An exaggerated inflamma&shy;tory response is present as evidenced by raised markers of inflammation such as CRP. The mortality rate has been re&shy;ported to be as high as 5.5% (Hacimus&shy;tafaoglu et al. 2004). Unlike lung ab&shy;scess, which is usually a solitary cavity showing rim enhancement and an air-fluid level on plain radiography or CT scan, necrotizing pneumonia manifests as multiple radiolucent foci (air crescent sign) that may be replaced by pneumato&shy;celes after the pneumonia (Hoffer et al. 1999). The underlying pathological pro-<br clear="all" />\r\n	cess involves ischemic necrosis due to vascular invasion by the organism with endarteritis causing nonviable or gan&shy;grenous lung tissue to separate from the parenchyma (Reich 1993), creating rims of lucency (Yella et al. 2005) and subse&shy;quently pneumatoceles as radiological findings (Hacimustafaoglu et al. 2004).</p>\r\n<p>\r\n	This is the first reported case of necro&shy;tizing pneumonia caused by <em>S. epidermi&shy;dis</em> in a neonate. We speculate that the invasiveness of this organism was heightened by the presence of risk fac&shy;tors such as an extrapleural catheter and the institution of steroid therapy. Early recognition of necrotizing pneumonia in a high risk infant presenting with cavitating lung lesions is important for rapid institu&shy;tion of optimal therapy to achieve a fa&shy;vorable outcome. In this case, aggressive antimicrobial therapy as conservative management instead of invasive percu&shy;taneous aspiration or drainage, resulted in complete resolution and avoided com&shy;plications such as pneumothorax and bronchopleural fistula.</p>\r\n', 1),
(432, 429, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Epilepsy is a chronic disorder character&shy;ized by a predisposition to recurrent un&shy;provoked seizures. Seizures are caused by abnormal neuronal discharges in the brain. Despite appropriate pharmaco&shy;therapy, about one third of newly treated epilepsy patients have refractoriness to medical treatment (Elger &amp; Schmidt 2008). The most prominent hypothesis for explaining refractoriness to antiepi&shy;leptic drugs (AEDs) in epilepsy is the multidrug-transporter hypothesis, which was first explored in chemotherapy-re&shy;sistant cancer. Excess efflux of AEDs across the blood-brain barrier (BBB) caused by overexpression of efflux transporters suggests that alterations in entry and extrusion of drugs into and out of cells may be an important mechanism of pharmacoresistance (Schmidt &amp; L&ouml;scher 2005). P-glycoprotein 170 (P-gp) is the most studied protein among the ATP-binding cassette (ABC) efflux trans&shy;porters (Schinkel et al. 1994), encoded by ABC subfamily B member 1 trans&shy;porter or multidrug resistance (ABCB1/ MDR1) gene. Evidence has shown that ABCB1/MDR1 expression was elevated in the epileptic foci of drug resistant epi&shy;lepsy up to ten times indi&shy;cating its main role in pharmacoresis&shy;tance (Tishler et al. 1995; Gottesman et al. 2002).</p>\r\n<p>\r\n	Different responses to pharmacother&shy;apy among patients with similar clinical phenotypes and taking the same doses of medication may be explained, in part, by single-nucleotide polymorphisms (SNPs) as the most frequent type of ge&shy;netic variation (Schmidt &amp; L&ouml;scher 2005). Numerous SNPs have been iden&shy;tified in ABCB1/MDR1 gene among which the C3435T variant is the most common polymorphism (Fung &amp; Gottesman 2009). The key question of this review is &ldquo;is the C3435T variant of the ABCB1/MDR1 gene associated with outcomes of AEDstreatment in epi&shy;lepsy?&rdquo; The initial phar&shy;macogenetic study demonstrated an as&shy;sociation of C3435T polymorphism with response to AEDs while the replication of this study has been controversial with consistent and conflicting studies. Herein, the sources of bias in association studies for the C3435T variant and refractoriness to antiepileptic drug (AED) therapy are briefly assessed.</p>\r\n<p>\r\n	<strong>P-gp Structure</strong></p>\r\n<p>\r\n	P-gp is the first discovered human ABC transporter more than 30 years ago in drug-resistant ovarian cells obtained from Chinese hamsters (Juliano &amp; Ling 1976).&nbsp; This protein is the product of the ABCB1/MDR1 gene, located at 7q21 and containing 28 exons with two transcrip&shy;tion start sites. It is a phosphorylated and glycosylated trans-membrane protein consisting of approximately 1280 amino acids and with a molecular mass of around 170 kDa. P-gp contains two ho&shy;mologous halves in which a short linker region joining the symmetric sequences together (Figure 1). Each half is com&shy;posed of one trans-membrane domain (TMD) containing six &alpha;-helix segments and one ATP-binding motif or ABC unit located at the cytoplasmic site. The two ATP-binding domains generate energy for interaction of drugs with drug-binding sites and for efflux transport function. P-gp mutations in TMDs 5, 6, 11 and 12 affect substrate specificity and indicate the presence of a drug-binding site in this region (Fung &amp; Gottesman 2009).</p>\r\n<p>\r\n	<strong>P-gp function</strong></p>\r\n<p>\r\n	P-gp functions as a transmembrane ef&shy;flux pump, by moving drugs from the in&shy;tracellular to the extracellular domain. It may also interact with drug molecules trapped within the cell membrane lipid bilayer. This protein is expressed in many normal tissues such as the liver, gastro&shy;intestinal tract, kidney, adrenal glands and the blood&ndash;brain barrier (BBB) (L&ouml;scher &amp; Potschka 2005a). Anatomic localization and ability of P-gp in trans&shy;porting a broad range of compounds suggest that this transporter acts as an effective cellular protector against toxic xenobiotics, drugs and metabolites that are its substrates. It does this by secret&shy;ing these compounds into the bile, urine and intestinal lumen, thereby preventing their accumulation in critical organs such as the brain. Altered P-gp expression and activity may influence an individual&#39;s sus&shy;ceptibility to drugs (L&ouml;scher &amp; Potschka 2005b). P-gp transports a broad spec&shy;trum of substrates in the plasma mem&shy;brane ranging in size from 250 to almost 4000 Da including many classes of drugs such as calcium channel blockers, lipid-lowering statins, opioids, chemothera&shy;peutic agents, HIV protease inhibitors, antibiotics, immunosuppres&shy;sive agents and b-adrenergic antago&shy;nists. Most AEDs are planar lipophilic agents and thus substrates for the P-gp transporter (Kwan &amp; Brodie 2005).</p>\r\n<p>\r\n	<strong>ABCB1/MDR1 polymorphisms</strong></p>\r\n<p>\r\n	Any two unrelated humans have millions of genetic differences caused by inser&shy;tion/deletions or SNPs. SNPs are the most abundant form of genetic variation. About 0.1% of an individual&rsquo;s DNA sequences are different among which ap&shy;proximately 80% is represented by SNPs. SNPs may be the causal factor of about 40% of the variation of the most common diseases and 70% of schizo&shy;phrenia (Goldstein &amp; Cavalleri 2005). Do the ABCB1/MDR1 variants account for the inter-individual variability in the phar&shy;macokinetics and pharmacodynam&shy;ics of drugs? The level of P-gp expres&shy;sion is highly variable between different indi&shy;viduals. Inter-individual variability of P-gp activity might affect blood levels and drug distribution to the specific target com&shy;partment. Insufficient or excessive drug penetration could cause adverse side effects or inactivity of drugs, respec&shy;tively (L&ouml;scher &amp; Potschka 2005a). The ABCB1/MDR1 gene is highly poly&shy;mor&shy;phic. Numerous SNPs have been found in the ABCB1/MDR1, the majority of which involve noncoding regions thus not affecting the P-gp amino acid se&shy;quence. More than 50 variants reside in the cod&shy;ing region which can possibly cause al&shy;tered function (Fung &amp; Gottesman 2009).</p>\r\n<p>\r\n	Allele frequency for most of the coding region SNPs is less than 8% in different ethnic populations with the exception of 3 SNPs in exons 12 (C1236CT), 21 (G 2677T/A), and 26 (C3435T) in ABCB1/MDR1 gene. Both synonymous C1236T and C3435T polymorphisms change a GGC codon to GGT (Gly412 Gly) and ATC to ATT (Ile1145Ile), re&shy;spectively (Table 1). So, what is the role of C3435T polymorphism in pharmacore&shy;sistance in epilepsy? An original report suggested a strong and significant asso&shy;ciation (P=0.006) between the CC geno&shy;type at 3435 in ABCB1/MDR1 gene and drug-resistant epilepsy (Siddiqui et al. 2003). Zimprich et al.&nbsp; (2004) confirmed the results of the first report (p = 0.035), but Tan et al. (2004a), with the exact repli&shy;cation of the first study and almost twice the sample size, failed to confirm the original findings (Zimprich et al. 2004; Tan et al. 2004a). Furthermore, Leschziner et al. (2008) in a comprehen&shy;sive genome wide approach failed to confirm the association of C3435T poly&shy;morphism and response to AEDs (Leschziner et al. 2008). In total, 26 studies attempted to examine this hy&shy;pothesis in epilepsy, but only 8 found a positive association (Table 2). The stud&shy;ies are divided into two groups: positive and negative associations, some of which are replications of the initial posi&shy;tive study (Table 2). It is unclear why these reports have found such contra&shy;dictory results. How can such contradic&shy;tory re&shy;sults be interpreted? Is there any obvious effect of C3435T polymorphism on re&shy;sponse to AEDs even if the pheno&shy;types are almost similar? There may be several reasons for such discordant findings which are discussed below.</p>\r\n<p>\r\n	<em>Publication bias</em></p>\r\n<p>\r\n	Authors and journal editors prefer to pub&shy;lish papers with positive findings which can lead to publication bias. Meth&shy;od&shy;ologic weaknesses and consequent bias or even robust study designs and analy&shy;ses with significant positive results at the 5% level in association studies may lead to false positive data probably arising by chance (Cardon &amp; Palmer 2003; Tan et al. 2004b). The initial positive associa&shy;tion study between C3435T polymor&shy;phism and multidrug resistance in epi&shy;lepsy (Siddiqui et al. 2003) is a publica&shy;tion bias and may be a false positive re&shy;sult. Hence 17 subsequent studies could not confirm the first finding by improving the statisti&shy;cal power through increasing sample size (Colhoun et al. 2003). To avoid such a false positive association report, it is rec&shy;ommended to publish the original study only with a= 0.005 or lower. Even at this significance level and 50% power, the chance of true positive findings is about 10%, and so the find&shy;ings should be inter&shy;preted with caution. In association stud&shy;ies between C3435T polymorphism and multidrug resistance in epilepsy, most authors have pointed to low power due to small sample size as a limitation. The total sample size in the 26 association studies is 7352 (282.77 &plusmn; 31.29), ranging from 45 to 609 epilepsy cases (Table 2). In order to have a power of 80%, these studies require a large sample size or collaboration between multi-centres and countries (Otto 2004).</p>\r\n<p>\r\n	<em>Definition of drug-responsiveness and drug-resistant epilepsy</em></p>\r\n<p>\r\n	There have been a number of definitions for drug-resistant epilepsy. Selection of the most appropriate definition for a par&shy;ticular study depends on the type of sei&shy;zure and epilepsy syndrome and the pur&shy;pose of using the definition. According to the National Association of Epilepsy Centres (NAEC), pharmacoresistance in epilepsy is defined when seizures do not come under control after 9 months of treatment under the care of a neurologist or when a patient does not become sei&shy;zure-free for 12 months during long-term state-of-the-art treatment with several suitable AEDs at maximal tolerated doses. Surgery, as an alternative to pharmacotherapy for about 5&ndash;10% of newly diagnosed patients can be used to define drug resistance because it would be assumed that the seizures remained uncontrolled after treatment with various drugs (Elger 2003; Schmidt &amp; L&ouml;scher 2005). Most association studies for C3435T and multidrug resistance epi&shy;lepsy have divided patients into &ldquo;drug-resistant&rdquo; and &ldquo;drug-responsive&rdquo; groups. The definition of drug-responsiveness and drug-resistance varies amongst these studies. In summary, drug-respon&shy;siveness is deﬁned as complete seizure freedom over three months to one year and/or more or 50% reduction or more in seizure frequency within one year, while drug-resistance is defined as having one or more seizures per month or four to 10 or more seizures in six months or one year, or less than 50% reduction of sei&shy;zures per year taking at least two AEDs, and also surgery for seizure control (Ta&shy;ble 2). Additionally, some studies have used healthy individuals instead of drug-responsive patients as controls (Kasperaviciute &amp; Sisodiya 2009). Since the definition of drug-responsiveness and drug-resistance is different between studies, patients who are classified as drug resistant in some studies may be drug-responsive in others. Therefore such variation in patients&rsquo; classification may lead to wrong conclusions. To have reliable findings, there need to be an agreement on the definition of drug re&shy;sponsiveness and drug resistance in epilepsy.</p>\r\n<p>\r\n	<em>AED-therapy </em></p>\r\n<p>\r\n	<em>a.&nbsp; AEDs&ndash;P-gp interactions </em></p>\r\n<p>\r\n	There is discrepancy in the transporter hypothesis as a mechanism for AED-re&shy;sistant epilepsy. Some reports suggest that P-gp plays a signiﬁcant role in medi&shy;ating resistance to AEDs. This includes drugs such as phenytoin (PHT), car&shy;bamazepine (CBZ), phenobarbitone (PHB), and lamotrigine (LTG) which, as P-gp substrates induce upregulation of P-gp expression in the brain. However, valproic acid (VPA) and vigabatrin do not produce the same effects (Sills et al. 2005). Therefore, based on this hypothe&shy;sis, in some association studies for ABCB1/MDR1 gene polymorphism and response to CBZ, VPA or other drugs that may not be substrates for P-gp, this polymorphism probably may not play any role in determining drug resistance (Kimura et al. 2007) and the patients, classified as drug resistant, may not really be pharmacoresistant. However, in-vitro observations do not support this hypothesis as CBZ, PHT, and VPA (Baltes et al. 2007a, Baltes et al. 2007b, Rivers et al. 2008), possibly belong to a class of &ldquo;weak substrates&rdquo; of human P-gp and may compete for drug-binding sites on P-gp and inhibit the transport of other substrates (Luna-Tort&oacute;s et al. 2008). Determination of which AEDs are real substrates of P-gp requires in vivo ex&shy;periments.</p>\r\n<p>\r\n	<em>b.&nbsp; AED&ndash;AED and AED-nonAED interactions </em></p>\r\n<p>\r\n	In patients who are on multiple drug ther&shy;apy, drug interactions may occur in which one drug may modify the activity of an&shy;other by enhancing or reducing its phar&shy;macologic effects with beneficial or harmful outcomes. AED&ndash;AED or AEDs and non-AED interactions in polytherapy regimen can significantly influence the clinical management in patients with epi&shy;lepsy, pharmacokinetically and pharma&shy;codynamically. For example, phenobar&shy;bitone (PHB), PHT, and CBZ as enzyme inducers, can increase the level of activi&shy;ties of various cytochrome P450 and UGT isoenzymes. Therefore, a combina&shy;tion of PHB, PHT, and CBZ with VPA, LTG, topiramate, and tiagabine in poly&shy;therapy increases and decreases their metabolism and half-lives, respectively (Patsalos et al. 2002). In association studies for C3435T polymorphism and refractory epilepsy, some authors have not differentiated between monotherapy and polytherapy treatments which could have influenced the results of their study. To prevent such a problem, the possible effect of ABCB1/MDR1 polymorphism on refractoriness to each AED monotherapy must be established.</p>\r\n<p>\r\n	<em>Haplotype </em></p>\r\n<p>\r\n	SNPs are often associated with particular variants existing on the same chromo&shy;some known as a &lsquo;haplotype&rsquo; which may play a role in response to drugs and dis&shy;ease susceptibility (Goldstein &amp; Cavalleri 2005). Such a non-random as&shy;sociation of SNPs is called linkage dise&shy;quilibrium (LD) (Goldstein &amp; Cavalleri 2005). Some reports have demonstrated significant LD between synonymous 3435C&gt;T and 1236C&gt;T and nonsynony&shy;mous 2677G&gt;T/A (Ala893Ser or Ala893Thr), suggesting that C3435T and 2677G&gt;T/A may influence P-gp function in epilepsy patients in response to AEDs (Kim et al. 2001). In association studies for C3435T polymorphism and refractory epilepsy with negative results, some au&shy;thors have suggested that since the C3435T variant is silent, its effect is pos&shy;sibly caused by specific haplotypes (CGC, TGC and TTT) of C1236T and G2677T/A (Hung et al. 2005). It was hy&shy;pothesized that the pres&shy;ence of the C3435T variant within a haplotype block can affect the timing of co-translational folding and P-gp struc&shy;ture which could influence its insertion into the membrane and subsequently change the structure of substrate and inhibitor interaction sites (Kimchi-Sarfaty et al. 2007). Haplotypes may potentially improve the statistical power under cer&shy;tain circumstances. However, the debate still exists whether SNPs or haplotypes are more appropri&shy;ate for genetic asso&shy;ciation studies (Tan et al. 2004b). Finally, many genes in&shy;cluding drug-transporters,drug-metabo&shy;lizing enzymes, and drug targets are re&shy;sponsible for drug respon&shy;siveness in epilepsy (L&ouml;scher et al. 2008) Therefore, one need to investigate the entire ge&shy;netic effect of these genes and their SNPs together, rather than study a par&shy;ticular variant or haplotype in a single gene.</p>\r\n<p>\r\n	<em>Population heterogeneity</em></p>\r\n<p>\r\n	<em>a.&nbsp; Epilepsy types</em></p>\r\n<p>\r\n	Epilepsy is heterogenous in nature, influ&shy;enced by different genes and environ&shy;ment. Drug resistance differ amongst patients with different types of seizures and epilepsy syndromes, and the popu&shy;lation (Duncan et al. 2006). A study on Finnish epilepsy cases performed by Sillanp&auml;&auml; et al. (1999) showed that pharmacoresistance to AEDs was seen inabout 78% of patients with sympto&shy;matic partial or symptomatic generalised epilepsies and 13% of patients with idio&shy;pathic generalised epilepsies but not in patients with idiopathic partial epilepsies (Sillanp&auml;&auml; et al. 1999). Such a high fre&shy;quency of drug resistance in sympto&shy;matic epilepsy (for example structural brain lesions) could be caused by over&shy;expres&shy;sion of P-gp due to the effect of frequent seizures or AEDs therapy (L&ouml;scher 2005c). This will release exci&shy;totoxic me&shy;tabolites from the altered tis&shy;sue and in&shy;hibit the efficacy of AEDs for treatment, irrespective of the C3434T gene poly&shy;morphism involvement (Schmidt &amp; L&ouml;scher 2005). Most asso&shy;ciation studies for C3434T gene poly&shy;morphism and re&shy;fractoriness have been conducted on either a speciﬁc subset of epilepsy types/syndromes or all the vari&shy;ous types of epilepsies which could therefore con&shy;found the results. To avoid such a bias, the relationship of pharma&shy;coresistance with C3434T gene poly&shy;morphism should be separately calcu&shy;lated for different epilepsy types including idiopathic, symptomatic and cryptogenic.</p>\r\n<p>\r\n	<em>b.&nbsp; Ethnicity effect</em></p>\r\n<p>\r\n	The distribution of allele and haplotype frequency is different among populations, reflected by differing ancestral histories. This difference may be caused by re&shy;sponses to natural selection, migration patterns, stochastic effects and founder events. Therefore, different ethnic groups may have various allele frequencies in both disease and non-disease genes (Cardon &amp; Palmer 2003). Genotyping of the C3435T polymorphism in different normal populations have shown that the C-variant is more frequent than T-variant and is the dominant ancestral allele, car&shy;ried by more than 74% Africans (Figure 2) (Ameyaw et al. 2001). The wild haplotype (CGC) frequency in South American and African populations is more than the mutated type (TTT) while the opposite is true for the Asians (Fung &amp; Gottesman 2009). The prevalence of the C-variant in the initial report was more in the drug-resistant than the drug-responsive Caucasian epilepsy patients (Siddiqui et al. 2003). However, in most of the studies with Asian cases (Seo et al. 2006; Kwan et al. 2007; Kim et al. 2008a, Kim et al. 2008b, Chen et al. 2007; Lakhan et al. 2009; Vahab et al. 2009; Szoeke et al. 2009) the opposite was seen, which was similar to the nor&shy;mal population (Figure 2), probably re&shy;flecting the complex role of C3435T polymorphism in the response of AEDs in different ethnic populations. In summary, based on the C allele and LD differences between various epilepsy and normal populations, inclusion of patients with various ethnicities in the association studies for C3435T variant and refractoriness in epilepsy can potentially introduce heterogeneity which would confound the findings (Tan et al. 2004a, Shahwan et al. 2007).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Despite more than 26 association studies in epilepsy involving the common ABCB1/MDR1 C3435T variant and re&shy;fractoriness, 16 of the replicated studies failed to confirm the original report. This review has provided an opportunity for better understanding of the methodologi&shy;cal problems in these association studies by highlighting five sources of bias which may confound the results. First, publica&shy;tion bias which can be caused by false positive results, found by chance in the initial report as a result of insufficient sample size, and low statistical power. Second, varying definitions of drug-re&shy;sponsive and drug-resistant epilepsy in the different studies could lead to differ&shy;ent results. Third, for some AEDs, it is not clear whether they are substrates of P-gp; hence justification of the results is difficult. Moreover, the interaction be&shy;tween AED&ndash;AED and AED-nonAED in polytherapy regimen can influence re&shy;sponse to AEDs. Fourth, certain haplo&shy;types, formed by C3435T with specific variants in ABCB1/MDR1 or other genes and SNPs may affect the response to AEDs. Finally, the studies may involve a heterogeneous population made up of combinations of various cases with dif&shy;ferent epilepsy syndromes or ethnicities. In order to obtain reliable results in the genetic association studies for C3435T polymorphism and refractoriness, these factors need to be controlled.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(433, 430, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The number of elderly people worldwide is growing rapidly. In 2000, there were 600 million people who were aged 60 and over and this number is expected to increase to 1.2 billion by 2025 and 2 bil&shy;lion by 2050, in which 80% are living in developing countries (Mujahid 2006). Data from the United Nations in 2002 showed that based on the 6 billion-world population, 7% of the world&rsquo;s population were 65 years and above, whilst those aged 60 years and above comprised another 10% (United Nations 2002). The elderly population in Malaysia is pro&shy;jected to increase from 6.6% in the year 2000 to 13.4% in 2025. Improved living conditions, better hygiene and advances in medical care have been reported as the reasons for a greater proportion of persons reach old age. However, as the population ages, there is an increased risk of this group developing systemic diseases like hypertension, heart dis&shy;ease, diabetes and chronic obstructive pulmonary diseases (World Health Or&shy;ganization 2003). As for oral health, re&shy;ports have revealed that the condition of this group is far from optimal. Their treatment needs are high due to eden&shy;tulism, missing teeth, caries, periodontal diseases and attrition resulting in im&shy;paired oral function, affecting quality of life (Petersen &amp; Yamamoto 2005). In Malaysia, report from the National Oral Health Survey of Adults 2000 (NOHSA 2000) stated that the prevalence of den&shy;tal caries and periodontal disease was still high among this group. The preva&shy;lence of dental caries for the age group of 65-74 years was 95.2% and DMFX of 23.2. More than one third of the popula&shy;tion in the group was edentulous, reach&shy;ing up to almost half of them edentulous by age more than 75 years, while for pe&shy;riodontal disease, the proportion of healthy periodontium decreased as age increased (National Oral Health Survey of Adults 2000).</p>\r\n<p>\r\n	&nbsp;As such, dental students need to be equipped with skills, knowledge and pos&shy;itive attitudes towards the elderly, so that they are more confident when providing dental care for this group (Nitscheke et al. 2004; Preshaw &amp; Mohammad 2005). In Faculty of Dentistry, UKMMC, geriatric dentistry is taught as part of a module which includes a series of lec&shy;tures and field trip activities to several old folks or nursing homes around Kuala Lumpur and state of Selangor. The series of lectures introduce students to basic aspects re&shy;lated to oral care and man&shy;agement of the elderly patient. At the end of the module, students are expected to demonstrate understanding of general health and oral health needs of the el&shy;derly and readiness to clinically manage an elderly patient.</p>\r\n<p>\r\n	In addition to clinical teaching, students are also exposed to the group of elderly in the community. Visiting the old folks&rsquo; or nursing home will provide opportunities for the students to meet and interact with the elderly in their own environment be&shy;sides in the clinic, to provide health edu&shy;cation in a community setting and to de&shy;velop a caring attitude towards under&shy;served communities.</p>\r\n<p>\r\n	Therefore, the main aim of this study was to assess the final year dental stu&shy;dents&rsquo; knowledge about older people, as well as their perception towards the training in geriatric dentistry. The findings may help to improve the conduct of ge&shy;riatric teaching in the faculty including the didactic courses, clinical rotation and field work, and to strengthen clinical man&shy;agement and communication skills of the students.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	All the final year students of Faculty of Dentistry UKMMC participated in the study (n=83) in 2007. They were required to complete a written questionnaire in the classroom that took between 10 to 15 minutes. The questionnaire comprised of three sections: 1) Palmore&rsquo;s Facts on Aging Quiz; 2) Oral health and social concerns of patients; and 3) Students&rsquo; perception towards the teaching of geria&shy;tric dentistry.</p>\r\n<p>\r\n	Palmore&rsquo;s Facts on Aging Quiz (FAQ II) (Palmore 1998), consisted of 25 items, used to measure the basic knowledge about elderly and aging, especially in the health-related professions (Table 1). The students were assessed whether the 25 item questions were true or false. Ques&shy;tions in Section 2 were based on the questionnaire developed by Jude et al in 2005 (Jude et al. 2005). The information obtained from this section was used to assess the students&rsquo; knowledge about specific psychosocial and health prob&shy;lems that can become barriers to the continuity of care. Questions in Section 3 were to assess the student&rsquo;s perception in the management and communication skills with the elderly patients before and after completing the geriatric dentistry module. Students were also required to select the main barriers and limitations faced during the conduct of the geriatric module in the faculty.</p>\r\n<p>\r\n	In establishing the validity of content, items in the questionnaire were ex&shy;amined and revised by our Dental Public Health consultant in Faculty of Dentistry, UKM. The questionnaire was pre-tested in a different group of clinical students prior to the actual study.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Demographic characteristic</em></p>\r\n<p>\r\n	A total of 83 final year students partici&shy;pated in the study. Their age were be&shy;tween 23 to 26 years old, with the mean age of 24.5 years. There were 14 male students (16.9%) and 69 female (83.1%). The ethnic group showed that majority of the students were Malays (63%; n=52), followed by Chinese (36%; n=30) and one Bumiputra-Kadazan.</p>\r\n<p>\r\n	<em>Knowledge on aging</em></p>\r\n<p>\r\n	Results from the Palmore&rsquo;s Facts on Ag&shy;ing Quiz (FAQ II) showed that 24.1% of the students (n=20) scored more than 70% correct answer for all items while 73.5% (n=61) scored between 50-70%. Based on individual items, the FAQ II items that fell within the high correct re&shy;sponse group were those that were re&shy;lated to the health of older adults (Table 1). These include knowledge on hyper&shy;tension, stress, physical strength, mem&shy;ory loss and osteoporosis. For moderate range of correct items, the issues were on clinical aspects like bladder capacity, clinical depression and constipation. Stu&shy;dents also had moderate knowledge on social aspects of aging like role of mod&shy;ern family members in taking care of the elderly (67.5%) and feeling of anxiety towards death among elderly (45.8%). The low category of correct response rate items mainly focused on behavioural aspects of the elderly. Less than 30% of the students answered correctly on items that included knowledge on health care practices between gender (26.5%), ability to change (24.1%) and practice of reli&shy;gion in old age (19.3) (Table 1).</p>\r\n<p>\r\n	<em>Awareness towards patients&rsquo; social concerns</em></p>\r\n<p>\r\n	The nine items that prevent older people from keeping appointment were ranked in order of students&rsquo; selection from the items (Table 2). The top three barriers were identified as: physical disability, transportation problems and complex and overlapping priorities (97.6%, 94% and 83.1% respectively). Students also per&shy;ceived financial barrier (79.5%) as an important factor that made the elderly missed their appointments compared to fear of the dental procedures (56.6%). More than half of the students also agreed that abuse or neglect by family members or caregivers was not the rea&shy;son for the elderly to miss their dental appointments.</p>\r\n<p>\r\n	<em>Students&rsquo; perception towards teaching of geriatric dentistry in UKM</em></p>\r\n<p>\r\n	Table 3 showed the results of students&rsquo; perception towards the teaching of geria&shy;tric dentistry in UKM prior and following the educational intervention. Comparison between both scores (pre-and post-tests)revealed very little changes on the per&shy;ceived understanding and abilities of the students upon completing the module. About 88% of the students reported that they had understood the general con&shy;cerns of the elderly before the interven&shy;tion, while 92% agreed during post-eval&shy;uation. After undergoing the geriatric module, the students also showed some increase in the percentage on what they perceived to be the barriers faced by older adults in getting dental treatment (88.0% and 98.7% respectively). Com&shy;munication was one of the barriers hig&shy;hlighted when managing the elderly pa&shy;tients. However, the results showed that the proportion of students who perceived to have overcome the barriers communi&shy;cating with the elderly did not change greatly after the intervention. Thirty-six percent of the students reported that they were still having problems in communi&shy;cating with the elderly after undergoing the module. No improvement was ob&shy;served on their perceived level of confi&shy;dence in management of older adults during dental treatment (pre- 72.2% and post- 69.3%).</p>\r\n<p>\r\n	As for the barriers and limitations faced during the module, the students ranked lack of educational materials specific to geriatric dentistry (80%) and lack of clini&shy;cal sites experience for geriatric patient care (80%) as the main source. Other barriers that were reported include lack of clearly articulated geriatric curriculum (72%), insufficient number of full-time faculty members (72%), lack of interest in the field (68%) and the overloaded dental curriculum (54.6%).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	One of the objectives of this study was to assess the knowledge of undergraduate dental students on aging. The assess&shy;ment was based on the Palmore&rsquo;s Facts Aging Quiz II (Palmore 1998), and the social concerns factors were based from the questionnaire developed by Jude et al in 2005 (Jude et al. 2005). The FAQ II has been widely used in the health-re&shy;lated disciplines to evaluate knowledge about the elderly. From this study, evalu&shy;ation of each individual item in the FAQ II showed that the high correct response group was those items which relate to the health of the elderly, while the moderate category were those items which were involved with clinical and social aspects of aging. These findings may reflect that the students had good to moderate knowledge about medical and general aspects of aging. Correct responses in the low category items that involve beha&shy;vioural aspects may reflect perceived insufficient knowledge about these is&shy;sues among the students. It is also im&shy;portant to note that dental students are often un&shy;der pressure to meet their edu&shy;cational and clinical requirements for graduation. Therefore, the need to em&shy;phasize certain issues in geriatric denti&shy;stry teaching like psychological and be&shy;havioural aspects among older people has to be looked into. Recently, geriatric dental pro&shy;grammes in the United States have fo&shy;cused on a broader range of top&shy;ics in the management of elderly pa&shy;tients. A survey conducted to 54 dental schools in the U.S showed that all schools taught about the barriers to dental care that may be experienced by elderly, while the psy&shy;chosocial problems and socioeconomic difficulties were taught in 96 and 94 per&shy;cent of schools, respectively (Mohammad et al. 2003). It appears that the geriatric dentistry teaching in many schools now has put more emphasis on social prob&shy;lems re&shy;lated to aging.</p>\r\n<p>\r\n	The second objective of this study was to determine the main social factors that prevent older adults from keeping to their dental appointments. By identifying the social concerns that may affect the utili&shy;zation of dental services by older people, dental students can now try to under&shy;stand the bio-psychosocial issues among this group and thus develop a positive attitude towards them. Similar to findings from this study, Fabiano et al. also re&shy;ported that transportation problem and complex and overlapping priorities were ranked high by the students as reasons why older adults failed their appoint&shy;ments. Abuse and neglect by family members were also ranked lowest (Fabiano et al. 2005).</p>\r\n<p>\r\n	About 79.5 percent of students in this study also perceived financial barrier as one of the important factors that made the elderly missed their appointments. This is true as several studies have re&shy;ported that income was a significant pre&shy;dictor of utili&shy;zation among older adults. Kiyak and Reichmuth (2005) examined some determinants of older person&rsquo;s dental service utilization and revealed that poor older adults were less likely to seek dental care compared to those in the high income group (Kiyak &amp; Reichmuth 2005). A similar pattern was also seen among the Chinese el&shy;derly whereby those who were better educated and wealthier had more visits to a dentist (Lo et al. 2001). However, further re&shy;search to look into the relation&shy;ship be&shy;tween perceived and actual rea&shy;sons why the elderly do not use the dental services or keep to their dental appointments need to be established.</p>\r\n<p>\r\n	This study also revealed that there were not many changes in the perceived competencies of managing older patients upon completing the geriatric module. Similar finding was observed in a study by Wood and Mulligan in 2000 that aimed to assess changes in the knowledge and attitude of dental students towards aging after receiving geriatric training. Overall, the study showed only a small increase in knowledge and unchanged attitudes toward elderly patients upon receiving the course (Wood &amp; Mulligan 2000). Therefore, additional research is needed to confirm whether this trend is actually true at the faculty level. Should it be con&shy;firmed, dental educators in the faculty need to identify strategies to improve the students&rsquo; competencies of managing older patients.</p>\r\n<p>\r\n	In the present study also, almost one-third of dental students were still unable to overcome barriers and did not gain more confidence in dental management of elderly patients. These findings were observed as the dental students were exposed to various types of elderly pa&shy;tients, with different level of complexity in relation to their clinical management. For example, some of the elderly patients presented in dental clinics would have multiple medical problems and impaired physical abilities. The number and type of patients treated in the polyclinic and the visits to multiple old folks&rsquo; home for each group would have some influence on and provide a variety of exposure for the stu&shy;dents. Therefore, the level of per&shy;ceived competencies about elderly indi&shy;viduals may vary due to the differences in clinical as well as community exposures. How&shy;ever, in a qualitative analysis follow&shy;ing a clinical rotation in long-term care facilities for the elderly, positive insights were ob&shy;tained from the dental students. They described the rotation as an important contributor to their clinical maturation, very worthwhile and enhanced their ap&shy;preciation of a dentist&rsquo;s professional re&shy;sponsibilities (MacEntee et al. 2005).</p>\r\n<p>\r\n	In the faculty, geriatric dentistry is taught within its module, and coordinated by the department of Dental Public Health. Exposures in community settings are greatly needed whereby the students can actually feel and experience the real life situation. Besides oral health, stu&shy;dents also obtain knowledge on other areas like behavioural sciences and quality of life. A rotation programme in outreach clinics where students will have the opportunity to treat and manage the oral health conditions of elderly patients is one of the strategies that can be un&shy;dertaken to improve geriatric teaching (Chavez &amp; LaBarre 2004). Therefore, collaboration with other clinical depart&shy;ments in the faculty is greatly needed. These departments can assist the stu&shy;dents by helping them to develop the skills needed (clinical and communication skills) when managing elderly patients. Then the training of geriatric dentistry will<br clear="all" />\r\n	not only focus on the techniques but also on other generic skills.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Future interventions should focus on im&shy;provingthe educational process, thus providing dentalstudents with positiveexperiences when dealing with el&shy;derly patients. This will then initiate the devel&shy;opment of positiveattitudes re&shy;garding the elderly among dentalprofes&shy;sionals.Early exposure to geriatric teaching for pre-clinical students may also be beneficial in the improvement of knowledge and per&shy;ception toward the elderly as they move to the clinical years.</p>\r\n', 1),
(434, 431, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The incidence of retropharyngeal ab&shy;scess (RPA) is rare with the advent and wide usage of potent antibiotics. On the other hand, the diagnosis could be diffi&shy;cult due to its masked clinical symptoms (Ameh 1999, Philpott et al. 2004). Retro&shy;pharyn&shy;geal abscess is common in chil&shy;dren, with peak incidence at the age of three to five years old (Philpott et al. 2004, Kamath et al.2007). Suppuration of retropharyngeal lymph nodes secondary to upper respi&shy;ratory infection is the main cause in pae&shy;diatric population (Kamath et al. 2007). In adults, it usually follows for&shy;eign body in&shy;gestion, trauma and in a rare occasion due to tuberculous infection of the cervi&shy;cal spine (Kamath et al. 2007, Goldenberg et al. 1997). Pak et al. (1999) have reported retropharyngeal abscess as a rare presentation in two patients with nasopharyngeal carcinoma.</p>\r\n<p>\r\n	Retropharyngeal abscess is a poten&shy;tially serious infection due to its compli&shy;cations which involves upper airway ob&shy;struction, internal jugular vein thrombo&shy;sis, mediastinitis, pericarditis, empyema, and carotid blow out (Pak et al. 1999, Craig &amp; Schunk 2003, Chao et al. 1999, Siou &amp; Yates 2000). Current mortality rate due to these complications accounts to 25% (Furst et al. 2000).</p>\r\n<p>\r\n	Patients with retropharyngeal abscess commonly present with neck pain, sore throat, fever, neck mass, respiratory dis&shy;tress or stridor and limitation of neck movement (Craig &amp; Schunk 2003). Physical examination may reveal bulging of the pharyngeal wall, pooling of saliva, swollen, and inflammed mucosa (Goldenberg et al. 1997).</p>\r\n<p>\r\n	Lateral neck radiograph may show loss of lordosis of the cervical spine, widening of the prevertebral soft tissue, and pres&shy;ence of air or foreign body in the soft tis&shy;sue (Goldenberg et al. 1997, Craig &amp; Schunk 2003, Boucher et al. 1999). Computer&shy;ized Tomography (CT) scan is the pre&shy;ferred imaging technique since it helps in identifying the extent of the le&shy;sion and its relation to the important structures in the neck. It is useful in dif&shy;ferentiating be&shy;tween cellulitis and ab&shy;scess(Boucher et al. 1999).</p>\r\n<p>\r\n	Treatment of retropharyngeal abscess consists of securing the airway, intrave&shy;nous antibiotics and intra oral incision and drainage (Goldenberg et al. 1997, Craig &amp; Schunk 2003).</p>\r\n<p>\r\n	The aim of this study was to review demographic patterns, clinical presenta&shy;tions and management of RPA<strong>.</strong></p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Operative records from 2000 to 2007 were retrospectively reviewed from two ENT referral centers in Malaysia. All cases of RPA were included. Eight pa&shy;tients were identified. Patients&rsquo; demo&shy;graphic information, presenting signs and symptoms were reviewed. Other findings which were reviewed are full blood count results especially the total white cell count, the culture and sensitivity results which were obtained intra-operatively, the imaging findings which includes lat&shy;eral neck x-ray and CT scan as well as the record of treatments which comprised of operative procedures and antibiotics.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	In our series, the patients were predomi&shy;nantly female. There were five females and three males. The median age was 47 years old. The age group and sex distri&shy;bution is illustrated in Table 1.</p>\r\n<p>\r\n	The presenting symptoms were varied and are summarized in Table 2. Most of the patients presented with dysphagia and or odynophagia, followed by short&shy;ness of breath with or without stridor, neck pain or swelling, restricted neck movement and hoarseness. Surprisingly, none of the patients had fever at the time of presentation.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Precipitating or predisposing factors are listed in Table 3. Four patients were di&shy;abetics. Being diabetics, these patients are prone to infection. Three patients had a history of fish bone inges&shy;tion. Among them was a 10 year old girl who was the youngest in the series.</p>\r\n<p>\r\n	Total white cell count was raised in all patients with predominantly neutrophilia. All patients underwent imaging studies. One patient underwent more than one im&shy;aging modalities i.e lateral neck radio&shy;graph and cervical contrast-enhanced computed to&shy;mography (CECT) scan which was sug&shy;gestive for retropharyngeal ab&shy;scess. Lat&shy;eral neck radiograph mostly showed wi&shy;dening of prevertebral soft tissue, air in the soft tissue, foreign body and loss of cer&shy;vical lordosis (Figure 1). CECT of neck confirmed the diagnosis and demonstrated ring enhancement and the extent of the abscess and its relation to important struc&shy;tures in the neck (Figure 2).</p>\r\n<p>\r\n	The patients were treated hydrated and medication was given to relief pain. Broad spectrum IV antibiotics were commenced based on most likely pathogens and surgeons preference. A combination of IV Rocephine and flagyl was used in one pa&shy;tient while IV cefuroxime and flagyl was administered to another pa&shy;tient. Other pa&shy;tients were treated with IV ampicillin and cavulanate. All patients underwent inci&shy;sion and drainage under general anes&shy;thesia.</p>\r\n<p>\r\n	Only two organisms were isolated from two patients in our study, i.e Klebsiella and Psuedomonas species, respectively. There were no growths in the rest of the patients. A Ziehl-Neelsen stain was per&shy;formed in all patients, for which none tested positive.</p>\r\n<p>\r\n	Two approaches were performed i.e transoral approach and cervical approach were treated with cervical drainage in view of the extension of the abscess lat&shy;erally to the parapharyngeal space. One pa&shy;tient had spontaneous rupture of the ab&shy;scess into the oral cavity while waiting for the operation.</p>\r\n<p>\r\n	Two patients who presented with air&shy;way obstruction underwent tracheostomy under local anesthesia. Tracheostomy tube were decanulated two days post-operatively. One patient underwent elec&shy;tive tracheostomy under general anes&shy;thesia for prolonged intubation secondary to septicaemia. Nasogastric tube was inserted in four patients who underwent intraoral drainage of abscess. The tube was kept for two days on average, post operatively.</p>\r\n<p>\r\n	All patients recovered well. Only one patient had septicaemia secondary to RPA and passed away on the thirteenth day of admission despite vigorous treat&shy;ment.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	RPA is common in children (Goldenberg et al. 1997).&nbsp; This is due to the fact that retropharyngeal nodes disappear by the age of five years. Most infections begin in the nose or nasopharynx, adenoids, and paranasal sinuses which drain to these nodes which later suppurate. There is a frequent history of an upper respiratory tract infection in children. In adults, retro&shy;pharyngeal abscess are caused by in&shy;strumentation, foreign body and trauma, and interestingly it is also a manifestation of tuberculosis of cervical spine (Kamath et al. 2007).</p>\r\n<p>\r\n	In our series, seven out of eight pa&shy;tients were adults and the only child was aged 10 years. RPA in this child was secondary to fish bone ingestion. Perfo&shy;ration of the posterior pharyngeal wall by the fish bone would have caused RPA in this patient. Four of them had diabetes and they were generally prone to infec&shy;tion.</p>\r\n<p>\r\n	Acute RPA is characterized by fever, dysphagia, difficulty in breathing, stridor and torticollis in which the neck becomes stiff and the head is kept extended. Pa&shy;tients with chronic RPA also complain of dysphagia and discomfort in the throat. How&shy;ever, these symptoms are less marked. In our series, most of the pa&shy;tients presented with ody&shy;nophagia and or dysphagia (75%), neck pain (37.5%) and difficulty in breathing or stridor (37.5%) as the abscess caused an obstruction to the airway and food pas&shy;sage. Examina&shy;tion commonly revealed bulging of post&shy;erior pharyngeal wall (Figure 3).</p>\r\n<p>\r\n	Sometimes, it is difficult to differentiate simple pharyngitis and RPA especially in the early stages of the disease whereby the signs and symptoms are not very prominent. We had one patient who was initially treated for pharyngitis. The pre&shy;senting complaints were mild odynopha&shy;gia and sore throat without fever. She was treated with oral antibiotics for a few days by her panel doctor.</p>\r\n<p>\r\n	Despite the administration of antibiotics, the symptoms persisted. She presented again with history of worsening sore throat and stridor without fever. She was intubated and blood investigation showed increased in total white cell count. CECT of neck was suggestive of RPA. The ab&shy;scess was drained via cer&shy;vical approach and Klebsiella species was isolated from the abscess. Unfortu&shy;nately, she devel&shy;oped septicaemia. A tracheostomy was performed due to prolonged intubation. However, despite the vigorous treatment, the patient passed away. These masked symptoms in RPA could be attributed to partially treated RPA, improper antibiotics and inade&shy;quate duration of treatment.</p>\r\n<p>\r\n	The use of antibiotics has modified the presentation of deep neck space infec&shy;tions. Most of the patients who presented with deep neck space abscesses had already received some form of antibiotic therapy as an outpatient. Signs and symptoms of infection could be masked.</p>\r\n<p>\r\n	This could result in missed or delayed diagnosis with development of complica&shy;tions.</p>\r\n<p>\r\n	Organisms which responsible in para&shy;pharyngeal abscess could be mixed aerobic and anaerobic. Among the aero&shy;bic organisms include streptococci, pre&shy;dominantly Streptococcus viridans and Beta-hemolytic streptococci (Page et al. 2008). Anaerobes are difficult to isolate and often are overlooked. Their exact role is difficult to ascertain from the medi&shy;cal literature because of the inconsistent methodologies used for their isolation and identification in many studies. Their isolation and identification requires ap&shy;propriate methods of collection, trans&shy;portation, and cultivation of specimens (Brook 2008). In our series, only two or&shy;ganisms were isolated from two patients, they were Klebsiella species and Pseu&shy;domonas species respectively. There was no growth in the rest of the patients. Anaerobe organisms could possibly be involved and as mentioned, it is difficult to detect such by culture and sensitivity study.</p>\r\n<p>\r\n	Soft tissue radiograph i.e. lateral neck radiograph in RPA is often diagnostic. It helps to localise the abscess in the neck and retropharynx (Flaup-Precaurariu et al. 2009). All radiographs must be in the true lateral position so that the preverte&shy;bral soft tissue could be measured cor&shy;rectly and must be obtained in maximum extension and in the inspiratory phase of respiration to prevent a false positive re&shy;sult. False positive radiographs com&shy;monly occur when an oblique view is obtained as well as during flexion of the neck in which the soft tissues bow into the airways. The lateral neck radiograph will demonstrate widening of preverte&shy;bral soft tissue, air in the soft tissue, for&shy;eign body and loss of cervical lordosis due to muscle spasm (Ameh 1999, Goldenberg et al. 1997). At the second cervical verte&shy;bral body level, soft tissue in excess of 7 mm in thickness is considered abnormal. At the sixth cer&shy;vical vertebra, tissue thicker than 22 mm in adults or 14 mm in children is also considered to be abnor&shy;mal. Sometimes, the widening of the pre&shy;vertebral soft tissue may also be due to retropharyngeal cel&shy;lulitis. Further evalua&shy;tions with CECT scan are useful in diffe&shy;rentiating an abscess from cellulitis (Goldenberg et al. 1997). It will show the extent of the abscess and its relation to important structures in the neck. Another non-invasive, inexpensive and reliable method is ultrasonography. It is capable in differentiating cellulitis from an ab&shy;scess (Chao et al. 1999).&nbsp; All of our pa&shy;tients underwent imaging studies. Sim&shy;ple lateral neck radiograph and CECT neck were found to be useful investiga&shy;tive tools in the diagnosis of RPA in our pa&shy;tients. If CT suggests presence of frank pus, we proceeded for surgical incision and drainage (I&amp;D) and if CT suggested a cellulitis we administered intravenous antibiotics therapy.</p>\r\n<p>\r\n	Most of the time, intubation is possible in patients with RPA. Intubation must be performed with great care to prevent rupture of the abscess and it is essential to have the head in a dependent position to prevent aspiration. However, in cases of difficult intubation due to the large swel&shy;ling of the posterior pharyngeal wall which obstructs the view of the laryngeal inlet, tra&shy;cheostomy is indicated. In our series, two patients underwent emer&shy;gency tra&shy;cheostomy under local anes&shy;thesia due to upper airway obstruction at the time of presentation. A tracheostomy was per&shy;formed to secure the airway in patients with large abscess.&nbsp;&nbsp;</p>\r\n<p>\r\n	Transoral incision and drainage to&shy;gether with intravenous antibiotics are the preferred treatments for retropharyn&shy;geal abscess (Ameh 1999, Craig &amp; Schunk. 2003).&nbsp; It is performed under general anaesthesia since aspiration un&shy;der local anaesthesia will worsen respi&shy;ratory obstruction (Ameh 1999). We used two criteria proposed by Kirse &amp; Roberson (2001) for safe transoral drainage. First, the abscess should be medial to the great vessels. Second, the abscess should be confined: there should not be free pus in the fascial spaces of the neck. If the lateral space is involved, cervical approach is required. For transoral drai&shy;nage, the patient is po&shy;sitioned as for adenoidectomy. In our se&shy;ries, three of the patients underwent cervical incision due to the large RPA which ex&shy;tended to the parapharyngeal space. All of our pa&shy;tients underwent adequate inci&shy;sion and drainage with IV anti&shy;biotics coverage. Except one mortality, all of them fully re&shy;covered.</p>\r\n<p>\r\n	In conclusion, retropharyngeal abscess is a serious deep neck space infection with lethal complications. The diagnosis needs to be excluded in patients pre&shy;senting with vague symptoms of sore throat and odynophagia without fever. Partially treated retropha&shy;ryngeal abscess will mask the symptoms. Thus, thorough assessment of the patient is mandatory. A correct diagnosis should be achieved so that adequate treatment can be given to prevent mortality.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(435, 432, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Today, infectious diseases, which used to wreak havoc in Europe till mid 19<sup>th</sup> cen&shy;tury, are being regarded as diseases of the underdeveloped world. In Western countries, infectious diseases and nutri&shy;tional problems no longer dominate infant mortality and morbidity. However, in de&shy;veloping countries the reverse was the case. Mortality attributable to communi&shy;cable diseases, maternal and perinatal conditions and nutritional deficiencies were reported to be more than five times as frequent in low and middle income countries as compared with high income countries (Alvarez-Dardet &amp; Ashton 2000). Also, the World Health Organiza&shy;tion (1998) reported that &ldquo;of more than 50 million deaths worldwide in 1997, about one-third were due to infectious and parasitic diseases such as acute respi&shy;ratory diseases, tuberculosis, diar&shy;rhoea, HIV/AIDS and malaria&rdquo;. These infectious diseases still remain leading causes of premature death among adults in the de&shy;veloping world (Lindgren et al. 2005).</p>\r\n<p>\r\n	The above facts, to some extent, un&shy;derscore the importance of infectious diseases in understanding morbidity in a developing country like Nigeria. Further&shy;more, with the emergence of new infec&shy;tious diseases which are resis&shy;tant to well established antimicrobials, and the HIV/AIDS scourge for which an effective cure is yet to be found, the need for greater attention to be paid to preven&shy;tive health care cannot be over stated (Huang 2004; Jones et al. 2008).</p>\r\n<p>\r\n	Several studies have shown that dis&shy;ease occurrence and distribution in a population was not a random event (Lindgren et al. 2005; Jha et al. 2002). The process of transmission of water re&shy;lated diseases have been found to be influenced by factors relating to the host, the individual, and the environment. Multi&ndash;level researches have been con&shy;ducted in recent times to describe envi&shy;ronmental (ecological) effects and indi&shy;vidual level effects on transmission of diseases. Most of them have implicated &ldquo;social class&rdquo; or &ldquo;socio&ndash;economic status&rdquo; in the epidemiology of infectious dis&shy;eases (Poortinga et al. 2008; Abayawardana &amp; Hussain 2002).</p>\r\n<p>\r\n	Egboka et al. (1989) observed that in Nigeria, &ldquo;outbreak of water-borne dis&shy;eases such as cholera, yellow fever, dysentery, diarrhoea, and guinea worm occur peri&shy;odically resulting in fatality&rdquo;. Guinea worm is reported to be wide&shy;spread in many rural districts throughout central and southern Nigeria. An esti&shy;mated 650,000 Nigerians suffered from guinea worm infections every year (Kinley 1989; Egboka 1989). Though considerable pro&shy;gress has been made since, but the problem still persists, albeit at a much lower level (Brieger et al. 1997).</p>\r\n<p>\r\n	In Anambra State, South&ndash;Eastern Nige&shy;ria, majority of the riverine communities lived in poverty and illiter&shy;acy, with their beliefs and social practices steeped in their traditional cultural belief sys&shy;tem. Also notable was the conspicuous ab&shy;sence of basic infrastructure and social amenities, especially water, roads, elec&shy;tricity and health facilities. The socio-economic and cultural environment ex&shy;posed these communities to greater risk of infectious diseases.</p>\r\n<p>\r\n	The environment has been implicated as an important factor contributing to morbidity. There was a widespread belief that environmental quality was a major determinant of health and well&ndash;being (Fenwick 2006; Osuafor 1997; Cairncross &amp; Feachem, 1983). The relationship between polluted drink&shy;ing water and spread of cholera was es&shy;tablished by John Snow in his study of the epidemiology of cholera in London, from 1848 to 1854 (Park 1997).</p>\r\n<p>\r\n	Water related diseases have been tra&shy;ditionally presented in four categories: (1) Waterborne, are faeco-orally transmit&shy;ted through contaminated water or food. Ex&shy;amples are cholera, typhoid, diar&shy;rhoeas, poliomyelitis etc. (2) Water&ndash;washed, are due to insufficient quantity of water used e.g. trachoma, scabies etc. (3) Water-based, the pathogen spends a part of its life cycle in a water snail or other aquatic animals, e.g. schistosomi&shy;asis and guinea worm. Lastly, (4) water-related insect vector, spread by insects which either breed in water or bit near water. e.g. malaria and onchocerciasis (Park 1997; Mera 1997).</p>\r\n<p>\r\n	Numerous studies have shown the re&shy;lationship between certain diseases and water pollution and contamination (Fenwick 2006). Other sig&shy;nificant variables linked to morbidity in&shy;clude socio-economic factors as meas&shy;ured in terms of per capita income, edu&shy;cation, housing, employment (Obermeyer 1993; Trinder et al. 2000); lifestyle (Evans et. al. 2000; Bitton &amp; Mckea, 2000); increased human intrusion into tropical forest for purpose of mining, farming, settlement, and tourism (Demany 1996); foreign trade and ex&shy;panded markets for imported foods, which occasionally contain bacterial or viral contamination (Demeny 1996; Mera 1997). Ethnicity is believed to be related to epidemiology of a number of diseases. This was due to the fact that ethnic back&shy;ground implied group homo&shy;geneity of diet, hygiene, education, religious and other customs that influ&shy;ence human health (Vallin &amp; Behm 1980). Age, sex and stress are also known to influence morbidity differentials (Ramalingaswani 1994; Miles 1991). Several studies have also associated certain diseases with seasonal and cli&shy;matic changes (Miles 1991; Kendall 1991).&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	There are a range of theories such as epidemiological transition, multi-factorial causation theory, socio&ndash;economic depri&shy;vation theory, epidemiological triad, Ajzen&rsquo;s theory of planned behaviour, health belief model, political economy of morbidity etc. that could be useful in ex&shy;plaining the character and pattern of morbidity in a community. The theories of multi-factorial causation, political econ&shy;omy and economic deprivation were con&shy;sidered most appropriate for explanation of morbidity differentials in this study. The multi-factorial causation recognizes that disease predisposition is influenced by a number of factors, which were linked to socio&ndash;economic status, cultural, genetic and environmental variables. Most of these factors are linked to man&rsquo;s life-style and behaviour patterns. The Economic Deprivation theory is applied here to look at morbidity from the point of view of par&shy;ticipants&rsquo; socio-economic status, which is mostly determined by societal forces. In Nigerian society, the social structure plays a primary role in social placement. Those at the lower social strata are likely to experience greater health problems. The position adopted in this study is that the morbidity differential is produced by the social system which creates social inequality and socio-economic depriva&shy;tion.</p>\r\n<p>\r\n	From the political economy perspective, people&rsquo;s beliefs and actions may play an important role on their health status. However, the socio-political economy plays a more primary and deterministic role. The theory suggests that &ldquo;the possi&shy;ble range of health statuses and health behaviours of individuals is limited by their location in the social and economic system. As a result, a significant change in the health behaviour of any group of individuals is seen to require altering the social and economic context within which they live&rdquo; (Minkler et al. 1995).&nbsp;</p>\r\n<p>\r\n	In this study, the focus was on water-related diseases generally, but with spe&shy;cific emphasis on those that were trans&shy;mitted through oral consumption of con&shy;taminated water. Specifically, this study tried to identify the frequency of water&ndash;borne diseases such as typhoid, cholera, diarrhoeal illnesses, dysentery, infectious hepatitis and poliomyelitis as a result of exposure to untreated or contaminated water in the communities studied. It also explored those socio-economic factors as well as cultural beliefs and practices that influence the susceptibility of the people to water borne diseases. It sought to identify differences within and between the communities studied in relation to the occurrence of the water borne diseases.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This was a retrospective, cross sectional survey on water-borne disease experi&shy;ences of riverine communities in three Local Government Areas (LGA) of Og&shy;baru, Anambra East and Anambra West in Anambra State, South-Eastern Nigeria. In this study morbidity was investigated from a multi-factorial causation perspec&shy;tive, with emphasis on the social, cul&shy;tural and economic background of the people. Six communities were sampled from the three LGAs studied. The com&shy;bined population of the six communities were projected by Nigerian Population Com&shy;mision (NPC 2000) at 87,636. A total sample of 1200 respondents repre&shy;sent&shy;ing 600 households was drawn from the six communities. The data collection process focused on heads of the house&shy;holds, using a set of structured question&shy;naire. In addition, 600 cases were re&shy;viewed from hospital records in the communities studied.</p>\r\n<p>\r\n	To ensure representativeness of the communities and households, a combi&shy;nation of the following sampling tech&shy;niques was adopted &ndash; quota, cluster, systematic random and simple random. Method of data analysis included fre&shy;quency distribution tables, percentages, and analysis of variance.</p>\r\n<p>\r\n	Ethics approval was sought and ob&shy;tained from the University of Nigeria eth&shy;ics committee.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Table 1 shows that 51.5% are females, while 48.5% are males. Over sixty per&shy;cent (67.6%) of the respondents were engaged in either farming or fishing. Only 11.4% were civil servants or profession&shy;als. This is a typical characteristic of rural communities. The respondents were pre&shy;dominantly Christians (61.5%), while 38.5% belonged to the African Traditional Religion. A greater percentage of the re&shy;spondents, i.e. 49.8% had no formal educa&shy;tion. This is followed by 34% with primary level education. The population can best be described as predominantly rural, illit&shy;erate and agricultural in nature. These facts influence their economic status, lifestyle and possibly their health status.</p>\r\n<p>\r\n	Typhoid and intestinal worms comprise 25.4% and 19.3% respectively. Preva&shy;lence of dysentery was also quite signifi&shy;cant (15.4%). Poliomyelitis had the least percentage frequency of 0.9% (Table 2).</p>\r\n<p>\r\n	Review of 100 cases randomly col&shy;lected from hospitals and/or health cen&shy;tres located within the six communities is presented in Table 3. Most of the health facilities were health centres and as such were not equipped to deal with serious health problems like infectious hepatitis, chol&shy;era, typhoid and so on. Most of such suspected cases were either re&shy;ferred out or were taken to hospitals out&shy;side the LGAs. The result generally re&shy;veals that water related diseases rank higher than other diseases. For instance, malaria, which is a water-related insect vector disease, ranked highest among all dis&shy;eases found in all the communities with over 50% occurrence. The next high ranking diseases were gastro&shy;enteritis and diarrhoea.</p>\r\n<p>\r\n	The analysis of variance was used to test the significance of difference ob&shy;served in the occurrence of WBDs in re&shy;lation to a range of variables.&nbsp; It was hy&shy;pothesised that prevalence of WBDs de&shy;creases as the level of education of household heads increases. A two-way ANOVA was used to test this hypothesis. The result gave an F value of 10.81 and 12.26 for prevalence and edu&shy;cation re&shy;spectively, which were signifi&shy;cant at a p value of &lt;0.001, indicating that there was a significant difference in the prevalence of WBDs in relation to level of education. The conclusion here was that prevalence of WBDs tends to reduce with increased level of education of the household heads.</p>\r\n<p>\r\n	It was hypothesized that some occupa&shy;tional groups experience higher preva&shy;lence of WBDs than others. This hy&shy;pothesis was tested using a two-way ANOVA, which gave F values of 7.00 for prevalence and 9.70 for occupation, with significance at P value &lt; 0.001. An ob&shy;servation of the disease distribution showed that the greatest disease burden was borne by those in farming/fishing occu&shy;pation, with 49.2%, followed by un&shy;skilled labours with 19.3%. The lowest burden of WBDs was found amongst those in the professional category with 1.8%.</p>\r\n<div>\r\n	<p>\r\n		Table 4 shows considerable variation in the distribution of WBDs amongst the communities. The highest morbidity bur&shy;den of 22.1% was found in Nzam, the headquarters of Anambra West Local Government Area. This is followed closely by Ogbakuba with 18.7%. On the other hand, Otuocha has the lowest bur&shy;den of 12.1%. The variation was mainly due to the source of water used, with riv&shy;ers and ponds being the major contribu&shy;tors.</p>\r\n	<p>\r\n		The Latin Square Design (a three-way ANOVA) was used to determine the level and significance of interaction between community, prevalence and water source available to the communities (Ta&shy;ble 5). Re&shy;sult of the analysis shows sig&shy;nificant dif&shy;ference in the prevalence of water-borne diseases at P=0.001 level of signifi&shy;cance in the general distribution of these diseases and also, with regard to the sources of water used. When in&shy;terac&shy;tion between different variables is meas&shy;ured, the result shows that there is a sig&shy;nificant difference between communities and the water they use as well as between prevalence of the different wa- ter-borne diseases and the water source available to the communities. The es&shy;sence of this Latin Square Design was to establish the points of interaction and the significance of such interactions between different variables.</p>\r\n	<div>\r\n		<p>\r\n			<strong>DISCUSSION</strong></p>\r\n		<p>\r\n			This study specifically relates toinfectious diseases that re&shy;sulted from domestic use of contami&shy;nated water. Water-borne dis&shy;eases were quite prevalent in Nigeria (Rooy 1987; Egboka 1989; Kinley 1989). Although the riverine people have more water in their environment than other communities living in the upland, most of the water available to them were pond, river, and stagnant lakes, which were subject to contamination and as such are not suitable for consumption. The streams were used for household chores, including bathing (especially in the river), washing of plates, clothes and also served as sources of drinking water. The result of this was that it led to frequent outbreaks of water-borne diseases, often reaching epidemic levels.</p>\r\n		<p>\r\n			Results of this study showed significant differen&shy;ces in the prevalence of water borne dis&shy;eases.&nbsp; This was found to vary with the level of education of the house&shy;hold heads, being higher among those with lower education level. There was also a significant variation in the preva&shy;lence with regard to the sources of water used in the households. There were also differ&shy;ences in the prevalence of WBDs ob&shy;served between the communities due to source of water available to them (Ta&shy;ble 4).</p>\r\n		<p>\r\n			The higher prevalence of WBDs amongst those in farming and fishing households could be explained by their poor living conditions and lifestyle. Farm&shy;ing and fishing tend to be done as a col&shy;lective household activity, exposing them to the same risk conditions. The epide&shy;miology is driven by the fact that they drink from the same source, using shared utensils. Moreover, the type of water commonly available to them in the farm was pond, river or streams, which were most often unfit for consumption.</p>\r\n		<p>\r\n			The study indicated some social and economic factors that influence the prevalence of WBDs. In the riverine ar&shy;eas the people are predominantly farm&shy;ers and fishermen. A good number en&shy;gaged in petty trading on agricultural produce. They lived in typical rural set&shy;tings, with very poor road networks most of which became impassable during rainy seasons. Also, the health facilities were inadequate, and most boreholes were broken down, with the few functional ones producing murky and undrinkable water. Due to widespread poverty, most of the people could not afford sanitary facilities, and this predisposed them to high risk of disease.</p>\r\n		<p>\r\n			Due to low level of education and poor awareness of water related health issues, prevalence of WBDs were high. How&shy;ever, above and beyond all these internal factors, it is important to recognise that these communities are located and oper&shy;ate within a wider socio-economic and political system which they have no influ&shy;ence over, but which determines their living condition. Explaining the morbidity among the people require wider under&shy;standing of the social-eco&shy;nomic and po&shy;litical forces. This is primar&shy;ily why this study is predicated on Multi&shy;factoral Cau&shy;sation theory, Economic Deprivation the&shy;ory, and Theory of Politi&shy;cal Economy, all of which calls for a broader consideration of factors beyond the cultural beliefs and practices of the people to avoid &lsquo;blaming the victim&rsquo; that characterises many socio-psychological theories such as the Health Belief Model. As Minkler et al. (1995) noted, &ldquo;the pos&shy;sible range of health statuses and health behaviours of indi&shy;viduals is limited by their location in the social and eco&shy;nomic system. As a result, a significant change in the health behav&shy;iour of any group of individuals is seen to require altering the social and economic context within which they live&rdquo;.</p>\r\n		<p>\r\n			The problems of social inequality and marginalisation with all its ramifications were quite glaring in these communities. If the sanitary conditions in the communi&shy;ties are allowed to continue as they were, the occurrence of serious outbreaks of cholera and increased prevalence of ty&shy;phoid, diarrhoea, dysentery, intestinal worms and other water related diseases would be inevitable.</p>\r\n		<p>\r\n			In view of the above discussions, it could be argued that the political econ&shy;omy plays a primary role in the determi&shy;nation of the health status of the people. The differences in the prevalence of wa&shy;ter borne diseases between groups within the communities, and between the communities are a function of the type of water available to them. While some of the communities had access to safe portable water, some did not. Our data revealed that the prevalence of WBDs was higher among households and communities with unsafe water source, compared with those that relied on safe portable water.</p>\r\n	</div>\r\n</div>\r\n', 1),
(436, 433, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Nursing staff have been identified to pos&shy;sess high level of work stress (Dickinson &amp; Wright 2008; Healy &amp; McKay 1999; Wang &amp; Patten 2001; Weinberg &amp; Creed 2000) given the nature of their work envi&shy;ronment. Common sources of stress among nursing staff include workload, leadership style, professional relation&shy;ships and emotional demands (McVickar 2003). Perception of stress is very sub&shy;jective and depends on the interaction between such individuals and his or her environment (McVickar 2003).&nbsp;</p>\r\n<p>\r\n	In Malaysia, stress among nursing staff is prevalent (Emilia &amp; Noor Hasim 2007; Majdah 1999; Rokiah 1994). Rokiah (1994) reported more than 49.5% of nurses in a public hospital in Kuala Lumpur had stressful experience related to workplace factors. A study among medical and surgical nurses in a teaching hospital in Kuala Lumpur reported work&shy;load as a major source of stress (Emilia &amp; Noor Hasim 2007).&nbsp;</p>\r\n<p>\r\n	Nursing staff working in the same dis&shy;cipline as well as different disciplines or different workplaces may face different stress level which depends on the inte&shy;raction of the multiple factors involved. Psychiatry and emergency medicine are among two highly stressed working envi&shy;ronments for nursing staff (Kilfedder et al. 2001; Yang et al. 2001). Psychiatric nursing staffs have to cope with emo&shy;tional burden handling patients who are mentally ill. They face aggression, abuse and emotional trauma such as suicide among the patients that they treated. On the other hand, nursing staff working in emergency medicine faced a different emotional burden of having to deal with emergency cases and death among pa&shy;tients that they see in casualty.&nbsp;</p>\r\n<p>\r\n	Stress among nursing staff has to be dealt efficiently since it may lead to vari&shy;ous emotional problems that may be af&shy;fecting work functioning. Work stress among the emergency nursing staff has been associated with Posttraumatic Stress Disorder symptoms but the causal nature of such relationship needs to be further established (Judith et al. 2003).</p>\r\n<p>\r\n	The subjective experience of stress is the balance between two processes. i.e. the process of primary appraisal to de&shy;termine the meaning of event and a process of secondary appraisal which takes place to assess one&rsquo;s coping abili&shy;ties (Taylor 1990). The big question asked is, how do nursing staff working in stressful environment adapt and cope with such stressors?&nbsp;</p>\r\n<p>\r\n	This study aimed to compare stress level among nursing staff working in dis&shy;ciplines of psychiatry and emergency medicine, identify common coping styles used and to determine the relationship between stress and coping styles among nursing staff in the above disciplines.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This was a cross-sectional, study ex&shy;amining coping styles and stress level among nursing staff working in two stressful disciplines i.e. psychiatry and emergency medicine in two public hos&shy;pitals in Klang Valley. A total of 106 nursing staffs were universally sampled from the psychiatric ward and the emer&shy;gency department.</p>\r\n<p>\r\n	Demographic data such as age, sex, race, marital status and duration of working at the current workplace were collected. Self-rated questionnaires were used and subjects were given two weeks to complete the questionnaires. Stress Arousal Checklist (SACL) (Cox &amp; Mackay 1985; Corcoran &amp; Fischer 2000) as&shy;sessed stress level whereas coping in&shy;ventory for Stressful Situations (CISS) (Endler &amp; Parker 1990) assessed coping styles among the subjects. The stress dimension of the SACL refers to the subjective experience in response to the external environment (e.g. tense, relaxed, apprehensive, worried) whereas the arousal dimension refers to the ongoing somatic or autonomic activity (e.g. ener&shy;getic, drowsy, stimulated, activated). The stress scores ranged from 0 to 18 whereas the arousal score ranged from 0 to 12, whereby higher scores reflected more stress and arousal.</p>\r\n<p>\r\n	The CISS is a 48 item questionnaire measuring coping styles i.e. task, emo&shy;tion, avoidance, distraction and social diversion coping. Nursing staff with good understanding of English and consented to participate were included. The study was approved by the ethical committee of the respective settings.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	The statistical package of Social Science was used to analyse the data. Indepen&shy;dent t-test, Chi-square test, correlation test, and ANOVA test were used to ana&shy;lyse the data.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 106 respondents participated in this study. The mean age for psychiatric nursing staff was 31.45<u>+</u> 53 whereas the mean age for casualty nursing staff was 28.62<u>+</u>5.36. There was a statistically significant difference in age between the two groups with the psychiatry nursing staff being significantly older than the casualty nursing staff. Other demo&shy;graphic data showed insignificant differ&shy;ence between the two groups (Table 1).</p>\r\n<p>\r\n	Stress level among the nursing staff in both disciplines was shown in Table 2. There were no significant differences in stress levels between the two groups.</p>\r\n<p>\r\n	The most commonly used coping style among the nursing staff in both groups was task coping and the least was social diversion. There were no significant dif&shy;ferences in terms of coping styles be&shy;tween the groups (Table 3).</p>\r\n<p>\r\n	Relationship between stress and coping styles were demonstrated in Table 4.&nbsp; Stress showed significant weak correla&shy;tions with task coping and emotion cop&shy;ing. For stress dimension, there was a statistically significant weak negative correlation with task coping. However, a statistically significant weak positive cor&shy;relation was found with emotion coping. In other words, the higher the stress level, signified lesser use of task coping but the more use of emotion coping.</p>\r\n<p>\r\n	A reverse was seen for the arousal di&shy;mension. There was a statistically signifi&shy;cant weak positive correlation with task coping suggesting more use of task cop&shy;ing in a higher level of arousal. There was a statistically significant weak nega&shy;tive correlation with emotion coping sug&shy;gesting the higher the arousal, the lesser use of emotion coping.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	In keeping with previous studies, both psychiatry and emergency medicine nursing staff reported similar stress in the two different disciplines of nursing (Kilfedder et al. 2001; Wang &amp; Patten2001; Watson et al. 2009). Many factors such as individual, work and organisa&shy;tional factors have been identified to contribute to nursing stress (McVickar 2003). A complex interaction exists be&shy;tween these multiple factors.</p>\r\n<p>\r\n	This study examined the relationship between stress and coping styles, a fac&shy;tor that can be intervened to help reduc&shy;ing stress level among nursing staff. The most commonly used coping style among the nursing staff in both groups was task coping whereas social diversion is the least used. It is unclear why the nursing group chooses task coping and not other coping styles. Coping styles are said to be influenced by both the&nbsp;&nbsp; genetic and environmental factors (Busjahn et al. 1999). It has been suggested that differ&shy;ent stressful situations may override indi&shy;vidual&rsquo;s preference of choice of coping styles (Endler &amp; Parker 1990). For in&shy;stance, work-related problem commonly lead to task coping whereas health-re&shy;lated problem leads to more emotion-focus coping (Yang et al. 2001). This may explain the choice of task coping among the nursing staff.</p>\r\n<p>\r\n	In relation to the two dimensions of stress, this study&nbsp; found significant weak negative correlation between stress di&shy;mension and task coping but positive correlation between emotion coping and stress dimension. In other words, the higher the stress level, the lesser use of task coping but more use of emotion coping. Possibly, in a highly stressful situation, it is easier for an individual to respond emotionally in order to reduce stress as compared to cognitively re&shy;structuring the problems to alter the stressful situation. Thus, in a highly stressful situations, emotion coping is more commonly used compared to task coping. Therefore, it is understandable that nurses working in stressful environ&shy;ment would be using more of emotion coping compared to task coping.</p>\r\n<p>\r\n	Interestingly, the finding is vice-versa with arousal dimension. There was a weak negative correlation between emo&shy;tion coping and arousal, but a positive correlation with task coping. The higher level of arousal, the more use of task coping but lesser use of emotion coping. This was difficult to explain. Possibly, coping style was not the primary factor in determining stress in this situation but other factors in the individual, environ&shy;ment or organization play more roles.</p>\r\n<p>\r\n	Why is work stress among the nursing staff important? It has been shown that stress related to work as well as from outside source, contributed to anxiety and depressive disorders among health staff (Weinberg &amp; Creed 2000). A Canadian National Population Health Survey found significant association be&shy;tween work stress and major depression (Wang &amp; Patten 2001).</p>\r\n<p>\r\n	Being an important constituent the medical team, nursing staff need to be physically and mentally fit. In certain situ&shy;ations, they are exposed more to the daily stress compared to other members of the medical team. For instance, psy&shy;chiatric nursing staffs are usually the first liner to handle aggressive patients before doctors arrive. They need to be physi&shy;cally and mentally stable to cope with daily work stress. Listening to patients&rsquo; emotional problem can also be mentally exhausting thus the need to be mentally fit themselves. Nursing staff that are not mentally healthy, may be more vulnera&shy;ble to develop counter-transference when handling patients&rsquo; emotional and psycho&shy;dynamic issues.</p>\r\n<p>\r\n	Mental health of the nursing staff de&shy;pends on the level and source of stress they experienced as well as their ways of coping (Pryjmachuk &amp; Richards 2007). Adaptive coping styles will help the nurses coping better with their daily stress. Various studies in Malaysia have found&nbsp;&nbsp; avoidance coping to be asso&shy;ciated more with poor mental health (Chong 2000).</p>\r\n<p>\r\n	While this study does not help in our understanding of choices of coping styles among the nursing staff, the importance of stress and coping styles among the nursing staff cannot be underestimated.</p>\r\n<p>\r\n	Nevertheless, this study has its own li&shy;mitations. Firstly, the cross-sectional de&shy;sign allows relationship between va&shy;riables to be identified at one point in time only. Secondly, the sample popula&shy;tion may not represent the nursing pop&shy;ulation in Malaysia due to the small sam&shy;ple size and limited to two hospitals in Klang Valley only. Finally, other con&shy;founders such as life-event, social sup&shy;port and personality were not controlled thus may give important impact on the findings. Future studies may need to im&shy;prove on the method to allow a better interpretation of findings to facilitate bet&shy;ter understanding and allow better inter&shy;vention in terms of stress among the nursing staff and their choices of coping styles.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In terms of stress level, there was no sig&shy;nificant difference found between the psychiatry and the casualty nursing groups. Task coping was the most com&shy;monly used coping styles among the nursing staff in this study. Significant re&shy;lationship was found between stress and task and emotion coping styles. Stress among nurses needs to be given more serious consideration and proper inter&shy;vention. Regular stress management program which include effective coping styles should be introduced in the workplace to minimize the problem.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The authors would like to acknowledge Universiti Kebangsaan Malaysia, Ministry of Health and the respective hospitals.</p>\r\n', 1),
(437, 434, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Antrochoanal polyp (ACP) is a maxillary sinus polyp with the pedicle attached to the inner wall of the maxillary sinus, emerging from the natural (or the acces&shy;sory) ostium, extending to the choanae and the nasopharynx through the nasal cavity. About 4% to 6% of all nasal pol&shy;yps are ACPs, which are more common in males than in females (Basu et al. 2001; Basak et al. 1998). ACPs are mac&shy;roscopically identical to typical polyps, but are not associated with an allergic aetiology. Patients usu&shy;ally present with unilateral or bilateral nasal obstruction. In the paediatric population, sleep distur&shy;bances and mouth breathing are com&shy;mon. In adults, snoring and headache may be the presenting symptom.</p>\r\n<p>\r\n	The history of ACP recognition dates back to the 18th century. Once, it was believed that the large polyps in the posterior choana and nasopharynx origi&shy;nate either from the lateral wall of the nasopharynx or from the posterior aspect of the vomer. In 1906, Killian was the first to address the true site of origin and to point out the close relation between ACP and the maxillary sinus. He was also the first to recognize the enlarged antralostium (being 1.7cm to 2cm in diameter) in his report of 21 pa&shy;tients with ACP. He initially advocated snare avulsion as the mode of treat&shy;ment.</p>\r\n<p>\r\n	Various surgical techniques were sub&shy;sequently proposed as alternative ef&shy;fec&shy;tive procedures, because simple avulsion of the polyp had a high risk of recurrence. To minimize postsurgical recurrence, it is essential to completely remove the antral portion of ACP, to&shy;gether with the base of its origin.</p>\r\n<p>\r\n	The use of powered instrumentation during endoscopic sinus surgery has recently been described as an effec&shy;tive technique for removing ACPs from the nasal cavity and for complete re&shy;moval of the antral portion (Hong et al. 2001; Yanagisawa et al. 2001; Gendeh et al. 2004). Endoscopic middle meatal an&shy;trostomy using 30, 70 and 120 de&shy;gree nasal endoscopes combined with trans&shy;canine microdebridement further en&shy;hances complete removal of the an&shy;tral portion of the ACP (Gendeh et al. 2004; El-Guindy &amp; Mansour 1994).</p>\r\n<p>\r\n	Forty patients with ACP who were treated in our hospital over a period of ten years were reviewed to observe the clinical presentation, associated rhi&shy;nological findings and the surgical mode of treatment for ACPs.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	We retrospectively reviewed the clinical presentations and operative records of patients with ACPs treated surgically over a period of ten years between May 1998 and April 2008 at the Department of Otorhinolaryngology, Universiti Kebang&shy;saan Malaysia, Kuala Lumpur. Clinical symptoms, associated rhinological find&shy;ings and the surgical procedure were re&shy;viewed.</p>\r\n<p>\r\n	Consent was taken after explaining the full nature of the surgery and the risks involved.&nbsp; After administration of general anesthesia, all patients were given local infiltration prior to the surgery. The main author (Gendeh) used diluted Naropine 2mg/ml for local injection (1cc of adrena&shy;line 1 in 80 000 is mixed with 9 cc of wa&shy;ter, then 1cc of this solution is mixed with 9 cc of Naropine, 2mg/ml). Transoral greater palatine block was given on the affected side. Nasal packing (mixture of 1cc of adrenaline 1 in 1000, 2cc of water and 2 cc of cocaine 10 %, soaked in &frac12; inch ribbon gauze) was applied for 20 minutes prior to the surgical procedure.</p>\r\n<p>\r\n	On removal of the nasal packing a ret&shy;rograde uncinectomy was performed using a 4mm, 30 degree HOPKINS tele&shy;scope, for good visualization. The unci&shy;nate process was identified and fractured using RHINOFORCE-STAMMBERGER antrum punch. The uncinate process was removed infero-superiorly and a moder&shy;ate-sized middle meatal antrostomy was performed. The ACP was removed by using powered instrumentation (microde&shy;brider 30, 70 and 120 degree) and a HEUWIESER antrum grasping forcep. The microdebrider was set to a speed of 1600 rpm in oscillating mode, and suction power was set at 180 mm Hg. Saline or gentamicin wash-outs were performed to detach the ACP. Unlike the Caldwell luc operation, the intact maxillary mucosa apart from the attachment of the pedicle was preserved during the powered in&shy;strumentation.</p>\r\n<p>\r\n	A transcanine microdebridement was performed if the ACP could not be com&shy;pletely removed by the above method.&nbsp; After local infiltration was given using the diluted naropine, a stab wound incision was performed above the gingivolabial sulcus at the canine fossa (Figure 1). Through the transcanine opening, the remaining antral portion of the ACP was removed using powered instruments with assistance of the 30, 70 or 120 degree nasoendoscope (Figure 2) and subse&shy;quently closed using absorbable sutures.</p>\r\n<p>\r\n	Upon completion of surgery, nasal packs (Merocel or Rapid Rhino), if re&shy;quired, were placed in the nasal cavity for a day. After removal of the nasal packs, daily alkaline nasal douche or sterimar nasal spray and oral antibiotics (clavu&shy;lanate-potentiated amoxicillin 225 mg to 625 mg bd) were prescribed for 10 days.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	From May 1998 to April 2008, 40 patients with ACPs underwent surgical excision at our centre. The median age of the pa&shy;tients was 37 years (10 to 62 years), and the male to female ratio was 1:1.4. Fol&shy;low-up ranged from 3 to 60 months (Me&shy;dian: 30 months). None of the patients had symptoms of allergy but one patient had concomitant bronchial asthma. Three patients had an oral mass that was visi&shy;ble without retraction of the uvula during oral examination.</p>\r\n<p>\r\n	<em>Clinical features</em></p>\r\n<p>\r\n	The most common clinical symptom was nasal obstruction (37 patients, 92.5%) followed by rhinorrhoea (45%), postnasal drip (35%) and snoring (22.5%). Other symptoms are as shown in Table 1.</p>\r\n<p>\r\n	<em>Associated rhinological findings</em></p>\r\n<p>\r\n	The most common observation was chronic sinusitis, noted in 20 patients (50%). Others are shown in Table 2.</p>\r\n<p>\r\n	<em>Surgical treatment</em></p>\r\n<p>\r\n	The commonest surgery was middle meatal antrostomy, performed on 35 pa&shy;tients (87.5%) (Table 3). Before the in troduction of powered instrumentation at our institution, seven patients (17.5%) had ACPs removed by nasal forceps un&shy;der endoscopic visualization. Since its introduction, ACPs were removed with powered instrumentation in 33 patients (82.5%). Among these, six patients (15%) underwent combined transcanine micro&shy;debridement for complete removal of the antral portion of the ACPs using 30, 70 and 120 degree rigid endoscopes. There were no complications noted in the group treated with powered instrumenta&shy;tion.</p>\r\n<div>\r\n	<p>\r\n		Complications were noted in one pa&shy;tient (2.5%) in the group where trucut forceps was used with endoscopic visu&shy;alization. This patient developed high-grade fever, purulent rhinorrhoea, facial pain and headache three weeks post surgery. All the symptoms subsided after aggressive and appropriate intravenous antibiotics, nasal toilet and decongestant therapy.</p>\r\n	<p>\r\n		Septoplasty was performed in two pa&shy;tients (5.0%). This procedure was per&shy;formed in view of the severely deviated nasal septum which caused nasal block&shy;age. In addition, one patient each had frontal sinustomy performed for frontal sinusitis and inferior turbinate reduction for bilateral inferior turbinate hypertrophy.</p>\r\n	<p>\r\n		No major complications were noted in both cases.</p>\r\n	<p>\r\n		Currently, our centre&rsquo;s practice is to use powered endoscopic middle meatal an&shy;trostomy with or without transcanine mi&shy;crodebridement for all new cases of ACPs. With wide middle meatal an&shy;trostomy, the inferomedial or laterally at&shy;tached pedicles of ACPs can be re&shy;moved via the antrostomy.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		Herniation of the hypertrophic maxillary sinus mucosa through the antral ostium protruding into the oropharynx is the characteristic appearance of ACPs. They are also known as Killian polyps which are rare and tend to be dumbbell-shaped with a constriction at the natural or ac&shy;cessory ostium of the sinus. They are more common in men than women and usually appear before the age of 40 years (Basu et al. 2001; Drake-Lee et al 1997). However, our study showed no significant sex predilection in 40 patients (17 males and 23 females) z = 3.615, P &gt; 0.01 with a median age of 37 years. A similar sex ratio was found in an earlier study by the same author (Gendeh et al. 2004). The aetiology of ACP remains un&shy;known. There is no correlation be&shy;tween ACP and allergy (Drake-Lee et al. 1997; Soh &amp; Tan 2000). There was only one patient who had concomitant bronchial asthma in our study.</p>\r\n	<p>\r\n		The most common symptom was nasal obstruction (92.5%), followed by rhinor&shy;rhoea (45%), post nasal drip (35%) and snoring (22.5%). The less common symptoms were headache (12.5%), hy&shy;posmia (10%), lump in throat (10%) and nasal discomfort (10%). Orvidas et al. 2001, noted nasal obstruction (100%), rhinorrhoea (48%), snoring (36%) and mouth breathing (32%) in their 25 pa&shy;tients with ACP.</p>\r\n	<p>\r\n		Epistaxis is an unusual manifestation of ACP. In such cases, angiofibroma must be excluded in the teenage male patient (Basak et al. 1998) and malignancy in adult patients. In our study, none of the patients had epistaxis as the presenting symptom. Nine of our patients com&shy;plained of snoring, two of whom had a significant history and examination that suggested obstructive symptoms. Venkatachalam et al. (1998) reported two cases of ACP with obstructive sleep ap&shy;noea treated with endoscopic intrana&shy;sal polypectomy and middle meatal an&shy;trostomy. A repeat sleep study four weeks post surgery revealed reversal of the disturbed sleep pattern.</p>\r\n	<p>\r\n		Twenty patients (50%) in our study had chronic sinusitis, 12 patients (30%) had maxillary and ethmoidal sinusitis and six had bilateral pansinusitis (15%). Unilat&shy;eral pansinusitis and maxillary sinusitis were noted in one patient each (2.5%). This is consistent with the study of Basak et al (1998), who reported that 50 % of patients had concommitant sinusitis.</p>\r\n	<p>\r\n		Many surgical options for the treatment of ACPs have been suggested to mini&shy;mize postoperative recurrence. The an&shy;tral portion of the ACP should be re&shy;moved completely. The conventional Caldwell-Luc approach has been advo&shy;cated as the most successful way to completely remove polypoidal tissue (Myers 1986). This procedure offers good exposure for removal of the antral portion of the ACP. However, it is associated with an increased risk of injury to the in&shy;fraorbital nerve, postoperative cheek swelling and a longer hospital stay. It is not the recommended treatment in chil&shy;dren because of disruption of dentition and facial growth (Myers 1986; Yanagisawa et al. 1998). None of our patients had the Caldwell-Luc surgery performed in this study.</p>\r\n	<p>\r\n		Endoscopic surgery is an effective treatment for ACP (Myers 1986; Yanagisawa et al. 1998; Guilherme et al. 2007). In our study, 35 patients (87.5%) underwent endoscopic middle meatal antrostomy. Of these, seven (17.5%) had the antral portion of the ACP removed through the middle meatal antrostomy using the HEUWIESER antrum grasping forceps. This technique was more difficult and time consuming compared to the microdebriders. It can also lead to un&shy;avoidable stripping of the normal mucosa from its origin. The other five patients had natural widening of ostium due to the ACP. Hence, middle meatal antrostomy was not performed in these patients.</p>\r\n	<p>\r\n		Powered instrumentation in the removal of ACPs reduces the limitations of con&shy;ventional endoscopic removal (Hong et al. 2001; Yanagisawa et al. 2001). The benefit of using the powered instruments is the ability to remove tissue precisely without the stripping of adjacent mucosa, thus reducing intraoperative bleeding, better visualization and shorter hospital stay (Yanagisawa et al. 2001). Specimen collected in the tracheal trap can be sent for histological diagnosis (Sato &amp; Nakashima 2000; Zweig et al. 2000; McGarry et al. 1997).</p>\r\n	<p>\r\n		During the operation, if the antral por&shy;tion is inferomedially or laterally based, and repeated antral washout fails to dis&shy;lodge the antral polyp, a transcanine ap&shy;proach can be used for complete removal using microdebriders with the aid of 30, 70 and 120 degree nasal endoscopes. Hong et al. (2001), suggest a similar ap&shy;proach using powered instrumentation. They reported an improvement rate of 96.4% (27/28 pa&shy;tients). Complications such as infraorbital hyperaesthesia and abnormal facial growth were not noted in this study. Four of them relapsed where the initial opera&shy;tion was performed else&shy;were in three pa&shy;tients and in one patient at our centre. Three of them underwent endoscopic polypectomy with middle meatal an&shy;trostomy and one under&shy;went powered endoscopic polypectomy with middle meatal antrostomy. The earli&shy;est recur&shy;rence was three months post op&shy;eration. One of the patients had simulta&shy;neous oc&shy;currence of an inverted papil&shy;loma which was the first case identified in our centre, hitherto unreported in the medical litera&shy;ture. Our study discovered one patient who presented with bilateral antrochoanal polyps. Routine history taking revealed that she was on oral contraceptive pills (OCP). Powered en&shy;doscopic polypectomy with middle meatal antrostomy was per&shy;formed and she was adviced to stop tak&shy;ing the OCP. Subse&shy;quent follow-up at four years showed no recurrence.</p>\r\n	<p>\r\n		Our approach is similar to the technique suggested by Hong et al. (2001), but we do not routinely use the transcanine ap&shy;proach. Thirty three of our patients (82.5%) under&shy;went powered endoscopic removal of ACPs. Among these, six had combined transcanine microdebridement for com&shy;plete removal of the antral portion of ACP. No significant complications were noted in all cases in which powered instrumentation was used. This is con&shy;sis&shy;tent with the study by Hong et al (2001) and other reports.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		ACP is a rare entity but should be sus&shy;pected in any patient with unilateral or bilateral nasal obstruction. A thorough history and examination are warranted in all patients for its diagnosis and man&shy;agement. Powered endoscopic intranasal polypectomy with or without transcanine microdebridation should be the key in the treatment of ACP as this technique en&shy;sures better visualization with more com&shy;plete dissection, preservation of normal adjacent mucosa and decrease the chance of recurrence.</p>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(438, 435, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The increasing number of incidents of violence in the workplace internationally has only recently been recognized as a serious safety and health hazard be&shy;cause of the many horrific incidents that have been reported in the media lately. (International Labor Organization 2000). Many people were aware of the exis&shy;tence of violence but only a few voiced out their concerns until the Inter&shy;national Labor Organization started pub&shy;lishing its reports about violence. Work&shy;place vio&shy;lence is a substantial contributor to occu&shy;pational injury and death and is increas&shy;ingly causing concern to govern&shy;ment bo&shy;dies, health and safety profes&shy;sionals, employers and employees in many countries. This is the second leading cause of fatal occupational injury in the United States (McDonald 2001). Ac&shy;cord&shy;ing to Summers (1999), for non&shy;fatal as&shy;saults, the highest risk occupa&shy;tions are those pertaining to service-ori&shy;ented or&shy;ganizations such as hospitals, nursing homes, residential facilities and social service agencies.</p>\r\n<p>\r\n	In the healthcare setting, workplace vi&shy;olence has brought to light the real threat that these personnel face in their daily working lives, and employees within the hospital setting are potential victims of any form of workplace violence by pa&shy;tients, visitors or colleagues, causing them to suffer from extreme mental and physical stress. Surveys carried out by healthcare staff, especially nurses worldwide, have demonstrated that vio&shy;lence in the healthcare workplace is widespread and nurses as front line caregivers are often involved in dealing with victims of violence and the perpe&shy;trators of violent acts (Worthington 2000).&nbsp; According to Jackson et al. (2002), the health sector is also facing serious prob&shy;lems in the recruitment of students into nursing, and new graduates into the workforce, and retaining experienced nurses especially in specialized areas such as critical care, emergency and mental health to name a few because of workplace violence.</p>\r\n<p>\r\n	Workplace violence in the healthcare setting in Malaysia is also a serious problem (Department of Statistics 2002) but there has been very limited amount of research done to bring this matter to light. Nurses are said to be more likely to sustain injuries at work compared to any other professional group and a substan&shy;tial number of these injuries are the result of assaults or aggression (Carter 2000).&nbsp; According to May &amp; Grubbs (2002), nurse assault or abuse reduces confi&shy;dence on the job, the ability to function and cope and has also brought about conflicts with patients, family members, visitors and co-workers. DelBel (2003) found that 42 per cent of nurses felt that verbal abuse had a great impact on the turnover of nurses while 36 per cent be&shy;lieved that it contributed to absenteeism to a great extent.&nbsp;</p>\r\n<p>\r\n	This study was carried out to determine the prevalence of workplace violence and to obtain more information on workplace violence experienced by nurses in the Universiti Kebangsaan Malaysia Medical Centre (UKMMC). It was also carried out to determine the category of perpetrators and the common types of workplace vio&shy;lence.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	The study was conducted in UKMMC over a three month period from January till March 2004, using the descriptive cross-sectional design.&nbsp; Approval to con&shy;duct the study was obtained from the Medical Research and Ethics Committee (Project Code No. FF/001/2004, dated 8<sup>th</sup> January, 2004).&nbsp;&nbsp;</p>\r\n<p>\r\n	The definition of workplace violence adopted in this study was according to the United States National Institute for Occupational Safety and Health (NIOSH) (1996), which was&nbsp; &ldquo;Any physical assault, threatening behavior or verbal abuse oc&shy;curring in the work setting,&nbsp; it includes but is not limited to beatings, stabbing, sui&shy;cides, shootings, rapes, near homicides, psychological trauma such as threats, obscene phone calls, an intimidating presence, and harassment of any nature such as being followed, sworn at or shouted at&rdquo; (NIOSH 1996).</p>\r\n<p>\r\n	Recruitment of participants was done through poster advertisements inviting all nurses who had experienced workplace violence during the study period to par&shy;ticipate and it was on a totally voluntary basis. Only nurses who had experi&shy;enced workplace violence during this three month period were allowed to par&shy;ticipate in this study. Confidentiality was main&shy;tained throughout the study. A total of 59 nurses participated in the study but only 55 questionnaires were completely ans&shy;wered and could be used. The re&shy;spon&shy;dents comprised of staff nurses, nursing students and male nurses. All 55 had experienced workplace violence from various disciplines and areas of work in UKMMC.</p>\r\n<p>\r\n	Data was collected via self prepared questionnaires which had been tested for validity and reliability. The Cronbach&rsquo;s Alpha obtained on the questionnaire was 0.872. The self administered question&shy;naire comprised two parts, Part A and Part B. Part A collected the personal data from the participants such as age, gen&shy;der, ethnic background, marital status, professional status, the place of work and years of experience. Part B collected the data about the violent inci&shy;dent itself such as when, where and what form of vio&shy;lence that had taken place and who was the perpetrator.&nbsp;</p>\r\n<p>\r\n	Data entry and analysis was done using the Statistical Package for Social Science (SPSS) software version 11.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	The total number of nursing staff in UKMMC at the time of the study was 1502 and of this 55 (3.7%) reported workplace violence experienced during the three month study period. Therefore, an average of 18.3 (1.2%) of the nursing staff had experienced workplace violence per month.</p>\r\n<p>\r\n	The majority of the victims were within the age group of 23-27 years (n=36 or 65.5%) and of Malay ethnicity (n=44 or 80.0%). This was followed by Indians (n=10 or 18.2%) and &lsquo;others&rsquo; (n=1 or 1.8%) who was a foreign nurse from In&shy;dia. There were no Chinese respon&shy;dents. The majority of the nurses who took part in this study were females (n=53 or 96.4%) and the largest group of partici&shy;pants were the staff nurses (n=45 or 81.8%) (Table 1).</p>\r\n<p>\r\n	According to place of work, the most respondents were from the surgical wards (n=11 or 20.0%) followed by the emergency department (n=10 or 18.2%) and critical care areas (n=10 or 18.2%). The medical wards and psychiatric de&shy;partment recorded the same number of respondents (n=9 or 16.4% each), fol&shy;lowed by the operating theatre (n=4 or 7.2%) and Obstetrics &amp; Gynecology De&shy;partment (n=2 or 3.6%). Forty-seven (85.5%) of the respondents had between 1-5 years, five (9.1%) 6-10 years, one (1.8%) 11-15 years and 2 (3.6%) more than or equal to 21 years of work expe&shy;rience (Table 2).</p>\r\n<p>\r\n	The largest number of violent incidents took place in the surgical areas (n=12 or 21.8%) followed by the Emergency De&shy;partment, Psychiatric Department and critical care areas each experiencing ten (18.2%) incidents. The Medical wards had eight (14.5%) incidents, Operating Theatre three (5.5%) and Obstetrics &amp; Gy&shy;necology two (3.6%) incidents. The total number of perpetrators was 64 of whom 26 (40.6%) were females and 38 (59.4%) were males. The greatest number of vio&shy;lent incidents perpetrated against nurses was by patients (n=26 or 40.6%) followed by patients&rsquo; relatives (n=24 or 37.5%).&nbsp; Eight (12.5%) were doctors (n=4), head nurses (n=2) and nurse managers (n=2). Four (6.3%) of the perpetrators were peers and two (3.1%) were non-relative visitors.</p>\r\n<p>\r\n	There were altogether 64 perpetrators for the 55 victims.&nbsp; Eight nurses had ex&shy;pe&shy;rienced violence from two perpetrators at one time and one nurse from three per&shy;petrators at the same time (Table 3).</p>\r\n<p>\r\n	The 55 respondents experienced different forms of violence 97 times during the study period. This is be&shy;cause some respondents experienced two or three forms of violence at the same time.</p>\r\n<p>\r\n	The most common form of violence was verbal abuse (n=31 or 31.9%) followed by verbal threat (n=23 or 23.7%). The most common form of physical violence was &lsquo;being pushed&rsquo; (n=15 or 15.5%); other forms included being &lsquo;slapped&rsquo; (n=2), &lsquo;pinched&rsquo; (n=4), &lsquo;scratched&rsquo; (n=3) and &lsquo;spat at&rsquo; (n=2), which totaled 11.3% (n=11); hit with an object (n=6 or 6.2%); kicked (n=3 or 3.1%); grabbed (n=3 or 3.1%); bitten (n=2 or 2.1%); sexually as&shy;saulted (stroked thigh) (n=2 or 2.1%) and strangled (n=1 or 1.0%). There were no incidents of being hit with a fist or cut by a knife (Table 4).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The main objective of this study was to determine the incidence of workplace violence in UKMMC, which to our know&shy;ledge has not been studied earlier. During the study period of three months (90 days), 55 nurses (3.7%) expe&shy;rienced workplace violence which trans&shy;lates to an average of 18.3 (1.2%) nurses per month and 0.6 nurses each day. This means that approximately one nurse is being abused every other day in UKMMC.&nbsp;</p>\r\n<p>\r\n	Forty-five (4.1%) out of 1091 staff nurses experienced workplace violence during the 90-day period. In other words, in every two days, one staff nurse is abused.</p>\r\n<p>\r\n	Therefore, according to the findings of this study, staff nurses experienced the most number of violent incidents. Anderson &amp; Stamper (2001) found that 90% of staff nurses experience at least one inci&shy;dent a year of abusive anger, conde&shy;s&shy;cension or being ignored by a physician and every two to three months 30% ex&shy;perience sexual abuse, ranging from lewd remarks to inappropriate touching.</p>\r\n<p>\r\n	The majority of the victims in the pre&shy;sent study were in the relatively younger age group of 23-27 years (36 or 65.5%) followed by the age group of 18-22 years (9 or 16.4%).&nbsp; Astrom et al. (2002), also found that more of the younger staff (less than 40 years) reported exposure to vio&shy;lence than older staff (more than 40 years). Female victims (53 or 96.4%) outnumbered the male victims (2 or 3.6%). This could be explained by the fact that in UKMMC there are only 43 male nurses compared to 1459 female nurses which gives a female to male ratio of 34:1. When the percentage is calcu&shy;lated by gender, 3.6% of the female nurses and 4.6% of the male nurses re&shy;ported violence during the study period.&nbsp;</p>\r\n<p>\r\n	The male nurses in UKMMC are mostly posted to the emer&shy;gency department and the psychiatric areas. This could be the main reason why the male nurses have reported more exposure to workplace violence. Senu&shy;zun &amp; Karadakovan (2005) indi&shy;cated that nurses working in these areas are more exposed to work&shy;place violence.</p>\r\n<p>\r\n	The majority (85.5%) of the respon&shy;dents who experienced workplace vio&shy;lence were those with 1-5 years of work experience. This may be because they are still new in their jobs and they are much less experienced. This observation was also made by Astrom et al. (2002), who found that inexperienced young staffs were most likely to be exposed to vio&shy;lence than those who had been work&shy;ing for a long time.</p>\r\n<p>\r\n	Our study revealed that workplace vio&shy;lence was widespread and not confined to any particular area. O&rsquo;Connell et al. (2000), also reported that violent inci&shy;dents are not confined to the commonly acknowledged high-risk areas such as the emergency and psychiatric settings but also occur at a high frequency in the general wards. In our study some of the perpetrators were peers and other pro&shy;fessionals, comprising colleagues such as doctors, nurse managers and head nurses. O&rsquo;Connell et al. (2000) found that medical staff and nurses themselves were one of the most frequent causes for the intimidation of nurses. Dalphond et al. (2000) and O&rsquo;Connell et al. (2000) found that patients and their relatives are the most common source of verbal and physical aggression to nurses, which was also a feature in our study.</p>\r\n<p>\r\n	Verbal abuse and threat was the most common form of violence in our study.&nbsp; Lee (2001) also reported that nurses ex&shy;perienced verbal abuse more commonly than physical violence. The types of physical violence in this study were simi&shy;lar with those reported by O&rsquo;Connell et al. (2000), who also found that physical vio&shy;lence included being chased, stabbed with scissors, being urinated or defecated on and having their hair pulled.</p>\r\n<p>\r\n	Our results were obtained only through those respondents who had willingly taken part in this study. It could not be confirmed that all the nurses who had experienced workplace violence during this study period had participated in this study. However, the low prevalence rate at UKMMC could be due to the general unwillingness of nurses to participate in this study.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	This study confirms that workplace vio&shy;lence remains an important issue among nurses, which calls for more effective measures to overcome this problem.&nbsp; It is also high time that nurses demand a safe working environment for themselves so that quality nursing care can be provided to patients. As mentioned earlier in rela&shy;tion to difficulty in retaining nurses in this profession, a safe working environment can be an encouraging factor for nurses to remain in this profession.&nbsp;</p>\r\n<p>\r\n	The main limitation of our study was that it relied only on voluntary reporting by the victims. The reported prevalence of violence is probably an underestima&shy;tion of the true violence prevalence.</p>\r\n', 1),
(439, 436, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	The World Health Organization currently classifies Langerhans cell derived neo&shy;plasms as Langerhans cell histiocytosis and Langerhans cell sarcoma (Jaffe et al. 2007). Langerhans cell histiocytosis is a proliferating disorder of Langerhans cells, whereas Langerhans cell sarcoma is a neoplastic proliferation of Langerhans cells that have overtly malignant cytologic features (Ben-Ezra et al.1991, Jaffe et al. 2007). First diagnosed in 1984 (Wood et al.1984),Langerhans cell sarcoma is a very rare disease that can present <em>de novo</em> or progress from an antecedent Langerhans cell histiocytosis (Lee et al.2006). The diagno&shy;sis relies on malignant cytological fea&shy;tures, a typical immuno&shy;phenotype that consistently shows ex&shy;pression of CD1a, S-100 protein, and Langerin (CD207), and the appearance of the typical Birbeck granules demon&shy;strated by electron mi&shy;croscopy (Ferringer et al. 2006, Jaffe et al. 2007). In practice, Birbeck granules could be dem&shy;onstrated only in a minority of cases due to the un&shy;availability of fresh tissues for ultrastruc&shy;tural analysis. Furthermore, cells may lose their ultrastructural characteris&shy;tics as they become less differentiated. In a re&shy;cent study, Verdijk <em>et al</em> (Verdijk et al. 2005)found lack of Birbeckgranules in Langerhans cells to be associated with mutation in the <em>Langerin</em> gene.</p>\r\n<p>\r\n	Currently, with the availability of antibo&shy;dies suitable for tissue immunohistoche&shy;mistry, the identification of the cell of ori&shy;gin of the different types of tumours can easily be performed on formalin-fixed paraffin-embedded tissues. In addition, the use of antigen retrieval techniques has allowed an expanded range of mole&shy;cules, previously detectable on fresh material only, to be studied in these tis&shy;sues. Immunohistochemistry on paraffin-embedded tissues now play a major role in the diagnosis of Langerhans cell sar&shy;coma, a diagnosis that has frequently been missed in the past.</p>\r\n<p>\r\n	In this report, we describe the histo&shy;morphological and immunohistochemical findings of a Langerhans cell sarcoma in a 31-year-old man in the absence of ul&shy;trastructural analysis.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	The patient was a 31-yr-old male who presented with localized back pain at the mid-thoracic region for two months. It was associated with poor appetite and weight loss. The disease was progres&shy;sive and had caused bilateral lower limb weakness and inability to walk. Past medical history showed that the patient had type 2 diabetes mellitus since he was 25 years of age.&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	A magnetic resonance imaging (MRI) scan revealed a pathological fracture of the eighth thoracic vertebra (T8) asso&shy;ciated with a heterogeneous lesion in&shy;volving the body and both pedicles, and narrowing of the spinal cord canal (Figure 1A and 1B). A computed tomographic (CT) scan guided biopsy of the lesion was performed and was sent for histo&shy;pathological examination.</p>\r\n<p>\r\n	Histologically, the biopsy showed frag&shy;ments of tissue infiltrated by aggregates of large cells with overtly malignant fea&shy;tures. The cells contained abundant eosinophilic cytoplasm with markedly pleomorphic nu&shy;clei, some displaying prominent nucleoli (Figure 2). Abnormal mitotic figures were frequently seen and many multinucleated giant cells were present. There were scat&shy;tered neutro&shy;phils and eosinophils in the background (Figure 2). Immunohisto&shy;chemical studies showed that the malig&shy;nant cells ex&shy;pressed CD1a, S100, CD68 and CD4 (Figure 2). The tu&shy;mour cells were nega&shy;tive for CD20, CD3, CD15, CD30, CK, MPO, HMB45 and Desmin. The antibod&shy;ies used and the results obtained are summa&shy;rized in Table 1. A combination of both the mor&shy;phological and immunohis&shy;tochemical findings supported the diag&shy;nosis of Langerhans cell sarcoma.</p>\r\n<p>\r\n	The patient underwent anterior and posterior instrumentation decompres&shy;sion and fusion procedures after which he symptomatically improved. He was sub&shy;sequently planned for radiotherapy.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Langerhans cell sarcoma is a rare tu&shy;mour and most previous reports have been in the form of case reports with paucity of infor&shy;mation from larger series of cases (Mardones et al. 2009, Takahara et al. 2009). Tani et al (Tani et al. 1992) sug&shy;gested that the diagnosis of malignant Langer&shy;hans cell neoplasm should be made on the basis of the pres&shy;ence of proliferation of Birbeck granule-containing tumor cells that have overt malignant cytological fea&shy;tures such as nuclear atypia and fre&shy;quent mitosis.</p>\r\n<p>\r\n	Ultrastructural analysis using electron microscope to demonstrate Birbeck granules in the cytoplasm of tumour&nbsp; cells however requires fresh tissue that are fixed in suitable fixatives such as glu&shy;taraldehye. Attempts at demonstrating Birbeck granules using formalin-fixed tissue samples were often unsuccessful (Lee et al. 2006, Zhao et al. 2009) due to fixation-related tissue ultrastructural damage. In the present case, the tissue sample was small and was entirely fixed in formalin and paraffin embedded, ren&shy;dering it un&shy;suitable for ultrastructural analysis. The diagnosis of Langerhans cell neoplasms had been previously made without the demonstration of Bir&shy;beck granules in the tumour cells (Ben-Ezra et al. 1991, Lee et al. 2006). Ben-Ezra et al. (1991) could demonstrate Bir&shy;beck granules in only three of the nine cases they reported.</p>\r\n<p>\r\n	Fortunately, with the advent of newer and better monoclonal antibodies for im&shy;munohistochemistry, a new marker (i.e., CD1a) that is specific for Langer&shy;hans cells is now available (Favara et al. 1997, Krenacs et al. 1993). CD1a repre&shy;sents one of the first phases of differen&shy;tiation of the CD1 antigen family. It is expressed on cell surfaces of cortical thymocytes and Langerhans cells (Krenacs et al. 1993), but it is not ex&shy;pressed on other non-neoplastic macro&shy;phages and den&shy;dritic cells such as inter&shy;digitating dendritic cells, follicular den&shy;dritic cells and plas&shy;macytoid dendritic cells (Jaffe et al. 2007). The narrow specificity of the anti&shy;body (Krenacs et al. 1993) and the ease of the immune stain&shy;ing process renders CD1a immunostain&shy;ing useful as a routine procedure for the diagnosis of Langer&shy;hans cell sarcoma (Nezelof &amp; Basset 1998).</p>\r\n<p>\r\n	In addition to CD1a, the tumour consis&shy;tently expresses S100 and Langerin (CD207). Interestingly, it can cause aber&shy;rant expression of the T cell marker CD4 (as shown in the present case) but the markers of the B and T cell lineages were absent. The immunophenotype of Langerhans cell sarcoma is identical to that of Langerhans cell histiocytosis (Jaffe et al. 2007). Pathologically, the differentiation between these two condi&shy;tions is purely morphological. Neverthe&shy;less, Langer&shy;hans cell sarcoma is associ&shy;ated with an aggressive clinical course and distant metastasis (Ben-Ezra et al. 1991, Misery et al. 2003, Tani et al. 1992) and should be considered as a separate entity from Langerhans cell histiocytosis (Ben-Ezra et al. 1991, Jaffe et al.&nbsp; 2007, Tani et al. 1992).</p>\r\n', 1),
(440, 437, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Rothmund-Thomson syndrome (RTS [MIM #268400]) is a rare and severe au&shy;tosomal recessive genodermatosis. It was first reported in 1868 in a patient with cataract and progressive poikilo&shy;derma by the German opthalmologist Rothmund and subsequently by the British dermatologist, Thomson with the current eponym introduced in 1957 (Taylor 1957). RTS is diagnosed clinically and the main features include proportio&shy;nate dwarfism, premature ageing, skin hyperpigmentation appearing within the first semester of life and longlife persist&shy;ing, poikiloderma, telangiectasia, brittle hair sometimes progressing to total alo&shy;pecia, dystrophic nails, juvenile cataract, photosensitivity and congenital skeletal defects as hypoplasia or absence of the radii and thumbs. There is a higher than expected incidence of cutaneous and non-cutaneous malignancies particularly osteosarcoma (Vennos et al. 1992, Wang et al. 2001). Several RTS patients have been described to have two malignancies including osteosarcoma and lymphoma (Wang et al. 2001).</p>\r\n<p>\r\n	Mutations in helicase RECQ4 gene are responsible for a subset of cases of RTS. The RECQ4 gene structure is unusual because it contains many small introns &lt;100bp. Wang et al. (2002) described a proband with RTS who had a novel 11-bp intronic deletion and showed that this mutation results in a 66-bp intron too small for proper slicing. Constraint on intron size may represent a general mu&shy;tational mechanism in RTS (Wang et al. 2002). This patient and his non-identical twin sister both had a homozygous muta&shy;tion of the RECQ4 gene which is respon&shy;sible for the severe phenotype asso&shy;ciated with RTS (Balraj et al. 2002). The twin sister developed osteosarcoma of the left humerus at the age of 15. She received chemotherapy and underwent limb salvage surgery. However, she de&shy;veloped metastases in her lungs and succumbed to the disease.</p>\r\n<p>\r\n	Response to radiotherapy in RTS pa&shy;tients has been variable. An abnormal DNA repair mechanism in some RTS patients predisposes them to malignancy and influence the tolerance of radiothe&shy;rapy. We are aware of three previous reports of patients with RTS developing squamous cell carcinoma (SCC) of the tongue (Borg et al. 1998, Martin-Bertolin et al. 1998, Dahele et al. 2004). A patient was reported to develop increased radio&shy;sensitivity and tissue intolerance to che&shy;motherapy (Borg et al. 1998) while another patient was reported to tolerate radiotherapy without evidence of in&shy;creased morbidity (Dahele et al. 2004). In this report, we describe a patient with a history of osteosarcoma who developed SCC of the base of the tongue. He un&shy;derwent a course of radiotherapy for the SCC of the tongue.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	The patient is a 27 year-old male, the first twin of a set of non-identical twins. The twins first presented with clinical features of RTS at the age of 10 months. Four generation pedigree showed a high de&shy;gree of consanguinity, and the parents are first cousins. Molecular analysis showed a homozygous mutation in RECQ4 helicase: 2746-2756-delTGGGCTGAGGC in IVS8 which is responsible for the severe phenotype associated with RTS in both twins, while both parents and the grandmothers were heterozygous. These findings were re&shy;ported previously by Balraj et al.(2002).</p>\r\n<p>\r\n	The patient developed multicentric os&shy;teosarcoma of the left humerus and right upper tibia at the age of 14 years. The tumor in the right upper tibia was re&shy;sected and allogenic bone grafting was performed. Subsequently, he received chemotherapy and later limb salvage surgery was performed for the tumor in the left humerus. However, he had to undergo an above knee amputation of the right leg as the site of the bone graft was infected with avascular necrosis and detachment of the proximal part of the bone graft. He did not receive radiothe&shy;rapy during the treatment for osteosar&shy;coma. He was disease-free for 10 years following therapy.</p>\r\n<p>\r\n	On follow-up he was noted to have right submandibular lymphadenopathy of 2.5cm x 2.0cm but defaulted referral for a biopsy. Three months later he was ad&shy;mitted for enlarging right neck swelling associated with right tongue pain and difficulty in eating. There was a history of intermittent fever, lethargy, and loss of weight (height 141.5cm, weight dropped from 26.3kg to 25.1kg, BMI dropped from 13.2 to 12.6). Physical examination re&shy;vealed a 3.0cm x 3.5cm ulcer at the right side of the posterior base of his tongue (Figure 1). Biopsy of the ulcer confirmed the presence of a well-differentiated SCC (Figure 2). It was staged as T2N2CM0 according to the 1998 American Joint Committee on Cancer (AJCC) staging system (American Joint Committee on Cancer, 1998).</p>\r\n<p>\r\n	Tumor excision was not performed in view of the extension of the tumor and his poor clinical condition. The patient was then advised for radiotherapy. In the first phase of radiotherapy the primary tumor and neck was treated to a total dose of 40 Gy in 20 fractions at 2.0 Gy per fraction. The patient developed se&shy;vere mucositis requiring regular intraven&shy;ous morphine, oral toilet and liquid nutri&shy;tional supplement and the treatment schedule was interrupted. After introduc&shy;tion of spinal cord shielding in the second phase of treatment, the primary tumor and neck was boosted with a further 26 Gy in 13 fractions. Subsequently the right neck was treated with a boost of 10 Gy in 5 fractions. The overall treatment time was extended to 63 days from a planned 42 days because of toxicity-related breaks and patient non-compliance. The tumor size reduced to approximately 0.5cm x 0.5cm at completion of radiothe&shy;rapy. The patient developed severe mu&shy;cositis with skin desquamation in the treated area (Figure 3) following treat&shy;ment. Local site infection was success&shy;fully treated with antibiotics. The toxicity symptoms subsided two months after com&shy;pletion of radiotherapy. Unfortunately,&nbsp; the tumor recurred and the patient died 16 months after diagnosis of SCC.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	We believe that in our patient, the severe phenotype was responsible for the mul&shy;tiple malignancies. In addition, being a chronic cigarette smoker increased his relative risk of SCC in the tongue. Can&shy;cers of the head and neck show a strong association with alcohol consumption and smoking, partic&shy;ularly cigarettes. This patient however did not have a history of alcoholism. The severe phenotype, chronic cigarette smoking, and previous chemotherapy for treatment of osteosar&shy;coma had most likely played a role in the pathogenesis of the tongue cancer in this patient.</p>\r\n<p>\r\n	In addition, we found that our patient had a severe toxicity from radiotherapy. A similar finding was reported by Borg et al. (1998) in a patient with an advanced tu&shy;mor who had greater than expected post&shy;operative radiotherapy toxicity, and the dose per fraction as well as the total dose was reduced (from 1.8 to 1.65 Gy per fraction and total dose from 66.6 Gy to 58.55 Gy). The treatment time in this re&shy;port was also extended from 45 to 71 days as a result of the toxicity. In contrast, Dahele et al. (2004) described a patient with SCC of the tongue who tolerated postoperative radiotherapy well, without evidence of increased early or late radia&shy;tion morbidity. The patient received ra&shy;diotherapy at a total dose of 50 Gy in 25 fractions over 5 weeks without undue tox&shy;icity. Given the heterogeneity in the syn&shy;drome, it is possible that patients&rsquo; sus&shy;cep&shy;tibility to radiation toxicity also varies. Re&shy;ports on abnormal DNA repair after irradi&shy;ation in RTS could explain for excess treatment toxicity in some pa&shy;tients.</p>\r\n<p>\r\n	Recently, Hicks et al. (2007) reported that the clinical behaviour of patients with RTS and osteosarcoma is similar to pa&shy;tients with sporadic osteosarcoma, and suggested that these patients should initially be treated with conventional doses of chemotherapy as prescribed by current protocols.However, five patients were reported to require chemotherapy dose modifications, most commonly due to mucositis from doxorubicin (Hicks et al. 2007). Therefore, careful clinical ob&shy;servation is needed to monitor for en&shy;hanced doxorubicin sensitivity in RTS patients. It is apparent that some RTS patients have abnormal DNA repair me&shy;chanisms that could lead to increased toxicity to radiotherapy and chemothe&shy;rapy. Therefore a complete screening for malignancies of RTS patients are impor&shy;tant to identify those patients who are prone to develop cancer and are there&shy;fore likely to experience increase treat&shy;ment morbidities. The present case illu&shy;strates the importance of regular follow-up of all RTS patients and screening those who are prone to develop cancer.</p>\r\n', 1),
(441, 438, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Metaplastic breast carcinomas (MBCs) encompass a heterogeneous group&nbsp; of breast cancers characterised histologi&shy;cally by intimate admixture of adenocar&shy;cinoma with dominant areas of spindle cell, squamous, and/or mesenchymal differentiation (Devilee &amp; Tavasoli 2003). The tumour may present in pure form of metaplastic spindle cell or squamous cell component without recognizable adeno&shy;carcinoma. It can be categorised into purely epithelial (when it is composed of squamous cell carcinoma only or adeno&shy;carcinoma and squamous cell) or mixed epithelial and mesenchymal e.g. carcino&shy;sarcoma. These tumours are rare, ac&shy;counting for less than 1% of all breast cancer cases with mean age of presen&shy;tation at 55 years old (Devilee &amp;Tavasoli&nbsp; 2003). Between 1984 to 2005, there were only 16 published reports of 505 cases discussing the clinical and pathological characteristics of this tumour (Beatty et al. 2006). Due to its rarity, data on the current understanding and management of metaplastic breast carcinomas are scanty. We report our experience in managing six cases of metaplastic breast carcinomas encountered at our centre over an 8-year study period. The histo&shy;pathological features and clinical out&shy;comes from these patients are reviewed.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	Cases were retrieved from the Breast Cancer Database at The Breast and En&shy;docrine Unit, Universiti Kebangsaan Ma&shy;laysia Medical Center (UKMMC). Infor&shy;mation on the demographics, diagnosis, tumour grade, oestrogen (ER) and progesterone (PR) receptors status, HER2 expression, clinical staging, treat&shy;ment, recurrence status and patient sur&shy;vival was recorded. Information on the patients&rsquo; survival details were obtained from the medical records and through telephone conversations with their family members. The patients were grouped according to tumour types; epithelial and biphasic histological subtypes. Disease free survival was defined as the time from diagnosis to distant recurrence (or death from cancer in the absence of re&shy;currence) or last follow-up. Overall sur&shy;vival was defined as the time from diag&shy;nosis to the time of death from the cancer or date of last contact for patients who are still alive or who had died from other causes.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 471 breast cancer patients were operated between 2000 and 2007 at our institution, of which six female pa&shy;tients were histologically confirmed to have MBCs, giving an incidence rate of 1.3%. Ethnically, the patients comprised four Malays and two Indians. The mean age of our patients was 51 years with the epithelial only group being slightly younger age (42 years) than the biphasic group (55 years). The mean tumour size was 9.5cm with the epithelial only group presenting with slightly bigger tumor size (13cm) compared to the biphasic group (8 cm). Histologicaly, three of the six pa&shy;tients had epithelial only tumours com&shy;posed of both adenocarcinoma and squamous cell carcinoma while the other three patients were diagnosed with tu&shy;mours of the biphasic type expressing both carcinoma and sarcomatoid compo&shy;nents. All patients had grade 3 tumours and all tumours were ER and PR negative. Only four patients had the HER2 status tested, all of which were negative. Axillary nodal involvement was found in all epithelial only group and one in the biphasic group.&nbsp; Four patients underwent mastectomy with axillary clearance, one patient had mastectomy only and one had wide local excision with axillary clearance. Two patients received neoadjuvant chemotherapy while another three had adjuvant chemotherapy. One patient refused chemotherapy. All of the patients had tumour recurrence with mean time of recurrence of 9 months. Five of them died two to three months after tumour recurrence. Table 1 summa&shy;rises the relevant clinicopathological findings.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Metaplastic breast carcinomas (MBCs) are rare types of breast cancer which are difficult to diagnose clinically as they do not have specific imaging patterns. They are also challenging to the pathologists as the tumours have a wide range of histo&shy;pathological features and the metaplastic area may dominate the whole tumour or occupy a very small area within the lesion. In this report, we identified six metaplas&shy;tic breast carcinomas and evaluated their clinicopathological parameters. We record an incidence rate of 1.3% which concurs with the reported incidence of 1-2% elsewhere (Roses 2005). Interes&shy;tingly, the six patients in our series were comprised of four Malay and two Indian ethnicity with no MBC diagnosed among Chinese patients. Similar ethnic differ&shy;ences in risks for MBCs was reported in an earlier study which reported a higher percentage of MBCs among Afri&shy;can Americans or Hispanics (Pezzi et al. 2007). The reason for this observation is unclear but probably suggests that racial or genetic factors may underlie the in&shy;creased risk for MBCs.</p>\r\n<p>\r\n	MBCs were thought to have either ori&shy;ginated as a single entity or clone that originates in the myoepithelial cells or its precursor, or those arising from pre-ex&shy;isting lesions such as papillomas, com&shy;plex sclerosing lesions and nipple ade&shy;noma (Denley et al. 2000; Gobbi et al. 2003; Popnikolov et al. 2003; Reis-Filho et al. 2003). This has been further sup&shy;ported by the fact that these neoplastic cells express vimentin, smooth muscle actin, muscle specific actin and p63 that are commonly associated with myoepi&shy;thelial cells (Schnitt &amp; Collins2008). Cyto&shy;keratin positivity is seen in the epi&shy;thelial and mesenchymal component of this tumour and it is thought that it is the metaplasia of the epithelial elements of a carcinoma that could have given rise to the sarcomatous changes (Saxena et al. 2004).</p>\r\n<p>\r\n	In most literatures, MBCs are ER and PR negative, as was the case in all pa&shy;tients reported here (Kuo et al. 2000; Khan et al. 2003; Luini et al. 2007; Pezzi et al. 2007). Similarly, most of the MBCs in our series did not express HER2 which is in keeping with a previous study (Barnes et al. 2005). The reported inci&shy;dences of lymph node involvement in these tumors were between 8%-56% (Kurian &amp; Al-Nafussi 2002; Khan et al. 2003). In this series of cases, axillary nodal involvement was found in the entire epithelial only group and only one of the patients in the biphasic group had nodal metastases. The distinctive lymph node negativity was observed in the presence of spindle cell component which may be due to the nature of sarcoma that tends to metastasize haematogenously rather than through lymphatic channels (Sneige et al. 2001; Gobbi et al. 2003). Despite the lower incidence of axillary lymph node involvement, these tumours had high metastatic potential either locally, to distant organs or both, in more than 50% of cases (Wargotz et al. 1989; Wargotz &amp; Norris 1989a; Rayson et al. 1999). Most distant metastases involve lung or bone (Wargotz et al. 1989; Wargotz &amp; Norris 1989b; Kurian &amp; Al-Nafussi 2002). In our series, we had five patients with distant metastases and one local recurrence (Table 1).</p>\r\n<p>\r\n	The average size of the tumours in this series was 9.5cm which was bigger than the usual size of infiltrating ductal carci&shy;nomas. This is most likely due to the ag&shy;gressive nature of the tumours with rapid growth rates. Most of these patients had poorly differentiated or undifferentiated tumours (compared to infiltrating ductal carcinoma) which will further lead to rapid growth rates (Pezzi et al. 2007). We were unable to do a statistical analysis on whether the epithelial only group tumours were larger than the biphasic group due to the limited number of cases. However, in this series the mean tumour size for the epithelial group was greater than the biphasic group. Five of our patients had mastectomy due to the large size of the tumours. Five of the patients had sys&shy;temic therapy (one refused) and most of them died within a year after surgery. In our series, the mean time of recurrence was 9 months and survival after recur&shy;rence was short. This concurs with other studies that showed that MBCs are less responsive to therapy with the conven&shy;tional regimes that are usually used for infiltrating ductal carcinoma (Wargotz &amp; Norris 1989a; Rayson et al. 1999). There is no survival advantage either with che&shy;motherapy or radiotherapy for distant metastases (Wargotz &amp; Norris 1989a). The median survival after metastases is 8 months after systemic treatment (Rayson et al. 1999). This means that the optimal systemic therapy for metaplastic breast carcinoma still needs further de&shy;fining.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Metaplastic breast carcinomas are rare types of breast cancers usually affecting the post menopausal age group. These tu&shy;mours have aggressive clinical beha&shy;viour, present with locally advanced dis&shy;ease, are large in size, high grade tu&shy;mours and triple negative. Despite treat&shy;ment with chemotherapy and radiothe&shy;rapy, the patients have a high risk of local recurrence and distant metastases which are potentially fatal.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(442, 439, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	The ageing process is the accumulation of oxidative damage to cells and tissues, which is associated with a progressive increase in the chance of morbidity and mortality (Beckman &amp; Ames 1998). Hu&shy;man ageing can be studied <em>in vitro</em>, spe&shy;cifically with normal human diploid fi&shy;broblasts (HDFs) which undergo a li&shy;mited number of cell divisions in culture and progressively reach a state of irre&shy;versible growth arrest, a process termed as rep&shy;licative senescence (Trougakos et al. 2006<em>)</em>. Senescent cells exhibit a gra&shy;dual loss of replicative potential that re&shy;sults in reduced harvest cell densities and cell saturation densities (Cristofalo et al. 1998) and expressed senescence as&shy;sociated &szlig;-galactosidase enzyme (SA b-gal) which can be detected by SA &szlig;-ga&shy;lactosidase staining (Dimri et al. 1995). Since senescent cells have been shown to accumulate with age in human tissues, it has been proposed that they contribute to organismal ageing (Campisi 2000).</p>\r\n<p>\r\n	HDFs offer a typical model for stress induced premature senescence (SIPS) as it exhibits a finite potential of differen&shy;tiation (Campisi et al. 2007). SIPS which shares the common features of replica&shy;tive senescence can be defined as sus&shy;tained effects of subcytotoxic stress on proliferative cell types, including irre&shy;versible growth arrest of the cell popula&shy;tion. Various oxidative agents such as hydrogen peroxide (H<sub>2</sub>O<sub>2</sub>) and ultraviolet light have been used to induce SIPS and H<sub>2</sub>O<sub>2 </sub>appeared to be the most commonly used agent.</p>\r\n<p>\r\n	Skin is the largest and outermost organ of the human body. Exposure to the envi&shy;ronment resulted in the cellular compo&shy;nents such as lipid, protein and DNA be&shy;ing targets to oxidative agents and sus&shy;ceptible to oxidative damage. Collagens are the main protein found in the skin of which collagen type I is the most abun&shy;dant. It is incorporated into collagen type III, which is an important substance in reticular tissues in the dermis while col&shy;lagen type IV is a crucial structure most commonly found in the basement mem&shy;branes and is responsible for the intrinsic cohesiveness of functional basement membranes (Varani 2006). Three distinct collagenases are responsible for de&shy;grading connective tissues namely MMPI, MMPII and MMPIII (Shingleton et al<em>.</em> 1996). MMPI is also called collage&shy;nase 1 or interstitial collagenase which degrades type I collagen while MMPII is known as gelatinase A and readily di&shy;gests the denatured collagens type I, II, III and gelatins. MMPIIIwhich is also known as stromelysin I, digests extra&shy;cellular matrix and activates proMMPI (Nagase &amp; Woessner 1999).</p>\r\n<p>\r\n	Skin tissue is frequently and directly exposed to a pro-oxidative environment, including ultraviolet radiation (UV), drugs and air pollutants. Besides external in&shy;ducers of oxidative attack, the skin has to cope with endogenous generation of reactive oxygen species (ROS) and other free radicals, which are continuously be&shy;ing produced and caused skin ageing (Suzanne et al. 2003).</p>\r\n<p>\r\n	Skin ageing is defined as structural changes of the skin as the age in&shy;creases. It is indicated as the inability to balance the important functions of the skin leading to cell death (Chandrasoma &amp; Taylor 2001). During the ageing process, there is an accumulation of oxi&shy;dation products such as oxidized pro&shy;teins, DNA adducts and lipid metabolites. In addition, there is a significant de&shy;crease in the defense system of the living cell, including a significant decrease in the antioxidant defense system (Beckman &amp; Ames 1998). Balance be&shy;tween oxidants and antioxidants are needed to minimize molecular, cellular and tissue damage. However, if the bal&shy;ance is upset in favour of the oxidants, oxidative stress could occur and results in oxidative damage. Reactive oxygen species (ROS) are known to cause oxid&shy;ative modification of DNA, proteins, lipids and small cellular molecules (Kang et al. 2005). To counteract the harmful effects of ROS, the various compartments of the skin are equipped with layer specific an&shy;tioxidant systems to maintain equilibrium between ROS and antioxidant systems and thus prevent oxidative stress (Thiele &amp; Ekanayake-Mudiyanselage 2007).</p>\r\n<p>\r\n	The unicellular algae <em>C. vulgaris</em> con&shy;tains many bioactive substances such as carotenoids, chlorophyll, toco&shy;pherols, ubiquinone, and protein. C.<em>vulgaris</em> is capable of inhibiting lipid peroxidation and interacts with enzymatic and non-enzymatic antioxidants system attenuat&shy;ing and resisting the oxidative stress and further lipid peroxidation. A recent study has shown that <em>C. vulgaris</em> has great potential as an anti&shy;oxidant with the ability to exert anti tumor effects against liver cancer (Mukti et al. 2009).</p>\r\n<p>\r\n	The ethanolic extract of <em>C. vulgaris</em> ex&shy;pressed significant antioxidant activity against naphthalene-induced oxidative stress in rat model (Vijayavel et al. 2007) while <em>Chlorella dichloromethane</em> extract ameli&shy;orates NO production and iNOS expres&shy;sion through the down-reg&shy;ulation of NF&kappa;B activity mediated by sup&shy;pressed oxidative stress in RAW 264-7 macro&shy;phage (Park et al. 2005). Another study reported that administration of <em>C. vulgaris</em> inhibited MMPI activity (Cherng &amp; Shih 2006).</p>\r\n<p>\r\n	Since skin tissue is frequently and di&shy;rectly exposed to a pro-oxidative envi&shy;ronment such as ultra violet radiation which results in oxidative damage and further causes cellular ageing, therefore in this study <em>C. vulgaris</em> was eva&shy;luated for its antioxidant properties in preventing skin ageing. Primary culture of HDFs which was exposed to H<sub>2</sub>O<sub>2</sub> was treated with <em>C. vulgaris</em> and the expression of <em>COL</em> and <em>MMP</em> genes which are involved in skin ageing was determined.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Cell culture, induction of senescence and treatment with C. vulgaris</em></p>\r\n<p>\r\n	Primary culture of HDFs was derived from circumcised foreskin of 9-12 year-old boys, from a local clinic. Written in&shy;formed consents were obtained from all subjects. The samples were aseptically collected and washed several times with 75% alcohol and phosphate buffered sa&shy;line (PBS) containing 1% antibiotic&ndash;anti&shy;mycotic (PAA, Austria). After removing the epidermis, the pure dermis was cut into small pieces and transferred into a falcon tube containing 0.03 % collage&shy;nase type I solution (Worthington Bio&shy;chemical Corporation, USA). Pure dermis was digested in an incubator shaker at 37&ordm;C for 6-12 hours. Then, cells were rinsed with PBS before being cultured in Dulbecco Modified Eagle Medium (DMEM) containing 10% fetal bovine se&shy;rum (FBS) (PAA, Austria) and 1% antibi&shy;otic&ndash;antimycotic at 37&ordm;C in a 5% CO<sub>2</sub> humidified incubator. After 5&ndash;6 days, the cultured HDFs were harvested by trypsi&shy;nization and culture-expand into new T25 culture flasks (Nunc, Denmark) with ex&shy;pansion degree of 1:4. When the sub&shy;cultures reached 80&ndash;90% confluence, serial passaging was done by trypsiniza&shy;tion and the number of population doublings (PDs) was monitored.</p>\r\n<p>\r\n	HDFs were divided into three treatment groups viz; i) untreated control&nbsp; ii) H<sub>2</sub>O<sub>2</sub>-induced oxidative stress, SIPS (10 &mu;M of H<sub>2</sub>O<sub>2</sub> exposure for two weeks) iii) cells pretreated with <em>C. vulgaris</em> extract before oxidative stress induction. Treatment with <em>C. vul&shy;garis </em>extract was started at pas&shy;sage 4 at dose 400 mg/ml based on the optimal cell viability that has been estab&shy;lished by the Department of Biochemi&shy;stry, Faculty of Medicine, Universiti Ke&shy;bangsaan Malaysia. It was a prolonged treatment whereby at passage 6, HDFs were in&shy;duced with H<sub>2</sub>O<sub>2 </sub>and treated with <em>C. vulgaris</em>extract.</p>\r\n<p>\r\n	<em>Preparation of C. vulgaris hot water extract </em></p>\r\n<p>\r\n	In this study, a green unicellular microal&shy;gae <em>C. vulgaris </em>Beijerinck grown in Bold&rsquo;s Basal Media was used. Dried <em>C. vulgaris </em>cells were suspended in distilled water at a concentration of 10% w/v, boiled at 100&deg;C for 20 minutes and then centri&shy;fuged at 10000 rpm for 20 minutes. The supernatant was subse&shy;quently lyophi&shy;lized to obtain <em>C. vulgaris </em>extract which was then added to cell culture growth medium as supple&shy;mentation.</p>\r\n<p>\r\n	<em>Morphology analysis and senescence-associated &beta;-galactosidase (SA-&beta;-gal) staining.</em></p>\r\n<p>\r\n	HDFs positive for SA-&beta;-gal activity was determined as described by Dimri et al. (1995). SA-&beta;-gal staining was performed with a senescent cell staining kit (Sigma, USA) according to the manufacturer&rsquo;s instructions. A total of 1X10<sup>5</sup> cells were seeded in six-well plates and incubated with fixation buffer (2% formalde&shy;hyde/0.2% glutaraldehyde) for 6-7 mins at room temperature. Cells were then rinsed three times with PBS and incu&shy;bated with 5-bromo-4-chloro-3-indolyl &beta;-D-galactopyranoside at 1 mg/ml in a buffer containing 40 mM citric acid/phosphate (pH 6.0), 5 mM K<sub>3</sub>FeCN<sub>6</sub>, 5 mM K<sub>4</sub>FeCN<sub>6</sub>, 150 mM NaCl, and 2 mM MgCl<sub>2</sub> for 4 h at 37<sup>o</sup>C in the absence of CO<sub>2</sub>. Blue staining was visible after incubation and the percentage of blue cells observed in 100 cells under a light microscope was calculated.</p>\r\n<p>\r\n	<em>Primer design</em></p>\r\n<p>\r\n	The expression level of each targeted gene was normalized to the housekeep&shy;ing gene which is GAPDH. Primer 3 software was used to design the primers for each targeted gene and GADPH which were further blasted with GenBank database sequences from National Center of Bio&shy;technology Information in order to obtain primers with high speci&shy;ficity. The se&shy;quence of the primers is shown in Table 1.</p>\r\n<p>\r\n	<em>Total RNA extraction</em></p>\r\n<p>\r\n	Total RNA from HDFs of each group was isolated using TRI Reagent (Molecular Research Centre, Inc.) according to the manufacturer&rsquo;s instruction. Polyacryl car&shy;rier was added to each extraction to pre&shy;cipitate the RNA and centrifuged for 8 mins at 12000g at 4&deg;C. The pellet was washed with 75% ethanol. After another centrifugation, the extracted RNA pellet was left to dry at room temperature for 15 mins and then dissolved in RNase and DNase free distilled water. Total RNA was stored at -80&deg;C immediately after extraction.</p>\r\n<p>\r\n	<em>Quantitative real-time RT-PCR</em></p>\r\n<p>\r\n	Gene expression level of <em>COLI, COLIII, COLIV, MMPI, MMPII, </em>and<em>MMPIII</em> was quantitatively analysed with one-step real time RT-PCR technique. The expression level of each targeted gene was then normalized to GAPDH. Real-time PCR reaction was performed with 100 ng of total RNA, 400 nM of each primer and iScript One-Step RT-PCR kit with SYBR Green (Biorad, Canada) according to the manufacturer&rsquo;s instruction. Reactions were run in Bio-Rad iQ5 with reaction profile as follows; cDNA synthesis for 20 mins at 50&deg;C; pre-denaturation for 4 mins at 95&deg;C; PCR amplification for 38 cycles with 10 secs at 95&deg;C, 30 secs at 61&deg;C. This was followed by a melt curve analy&shy;sis to determine the reaction specificity. Agarose gel electrophoresis was per&shy;formed for confirmation of the PCR prod&shy;ucts.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	Each experiment was carried out in dup&shy;licates. Data were reported as mean &plusmn; SD. Comparison between groups was made by Student t-test (two-tailed). p&lt;0.05 was considered statistically sig&shy;nificant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Cell morphology and senescence-associated &beta;-galactosidase expression.</em></p>\r\n<p>\r\n	Morphological changes were observed with ageing of the HDFs. Young HDFs displayed the normal spindle shape cha&shy;racteristic of fibroblast cells. However, in SIPS, the original fibroblastic shape was lost and HDFs became larger and flat&shy;tened with accumulation of cytoplasmic granular inclusions. Similar cell morphol&shy;ogy was observed for SIPS cells which were treated with <em>C. vulgaris </em>(Figure 1).</p>\r\n<p>\r\n	A positive blue stain of SA-&beta;-galap&shy;peared mainly in H<sub>2</sub>O<sub>2</sub>-treated HDFs (SIPS cells) suggesting the presence of senescent cells. Quantitative analysis showed the percentage of cells positive for SA-&beta;-gal staining was increased (p&lt;0.05) in SIPS compared to young un&shy;treated HDFs (Figure 2). Similar results were observed for senescent cells at passage 30 and SIPS treated with <em>C. vulgaris </em>(p&lt;0.05).</p>\r\n<p>\r\n	<em>Specificity of primers and real-time RT-PCR</em></p>\r\n<p>\r\n	Six senescence-associated gene primers were designed. Agarose gel electropho&shy;resis showed that each PCR product ap&shy;peared as single bands (Figure 3). The melting curve analysis showed a single and narrow peak of each PCR product (Figure 4) indicating that the primers de&shy;signed and the real-time RT-PCR proto&shy;cols were specific.</p>\r\n<p>\r\n	<em>Expression of COL and MMP genes</em></p>\r\n<p>\r\n	Treatment with H<sub>2</sub>O<sub>2</sub> caused a downre&shy;gulation of <em>COLI</em> and <em>COLIII</em> genes (Fig&shy;ure 5) and upregulation of <em>MMPI</em> gene (Figure 6) compared to untreated control HDFs (p&lt;0.05). H<sub>2</sub>O<sub>2</sub>-treated HDFs (SIPS cells) which were incubated with <em>C. vulgaris</em> extract showed downregula&shy;tion of <em>COLI</em> gene and upre&shy;gulation of <em>MMPI</em> gene compared to un&shy;treated con&shy;trol HDFs (p&lt;0.05). <em>COLIII </em>gene was upregulated with <em>C. vul&shy;garis</em> treatment compared to H<sub>2</sub>O<sub>2</sub>-treated HDFs (p&lt;0.05).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The present study evaluated the effects of <em>C. vulgaris</em> extract in possibly mod&shy;ulating H<sub>2</sub>O<sub>2</sub>-induced cellular senes&shy;cence (SIPS) in skin HDFs. Our results showed that when HDFs reached senes&shy;cence, there were clear changes in cell mor&shy;phology, decreased cell proliferation and increased senescence associated &beta;-ga&shy;lactosidase activity. Morphological changes are typical features of senes&shy;cent phenotype that can occur at both cellular and organism level (Cho et al. 2004). Senescent HDFs showed mor&shy;phological changes of flattened and enlarged cell shapes. This shift is accom&shy;panied by changes in nuclear structure, gene expression, protein processing and metabolism (Campisi 2000).</p>\r\n<p>\r\n	Bayreuther et al. (1988) stated that progressive morphological changes oc&shy;cur while cells aged <em>in vitro</em>. Senescent cells are bigger and a senescent popula&shy;tion has more diverse morphotypes than cells at earlier cumulative population doublings. In fact, a confluent senescent culture has a smaller cellular density than a confluent young culture, though this also occurs because senescent cells are more sensitive to cell-cell contact inhibi&shy;tion. These changes are concomitant with increased activity of &beta;-galactosidase measured at pH 6 (Dimri et al. 1995; Krishna et al. 1999). In the present study, <em>C. vulgaris</em> extract however was not able to show any morphological effects on H<sub>2</sub>O<sub>2</sub>-induced cellular senes&shy;cence.</p>\r\n<p>\r\n	Extracts of <em>C. vulgaris</em> has pre&shy;viously been reported to show antitumour effect in fibrobsarcomas (Konishi et al. 1985; Hasegawa et al. 2002) and liver can&shy;cer (Mukti et al. 2009). The algal ex&shy;tract has also been shown to exhibit possible anti&shy;cancer properties in hepato&shy;cellular carci&shy;noma cell lines, HepG2 (Llovet et al. 2003; Md. Saad et al. 2006). There was also a report of using <em>C. vulgaris </em>as a potential thera&shy;peutic agent against ad&shy;vanced glycation end product (AGE), in which AGE has long been rec&shy;ognized and implicated in the develop&shy;ment of age related disorders such as atherosclerosis and diabetes (Yamagishi et al. 2005). <em>C. vulgaris </em>also had been shown to mod&shy;ulate H<sub>2</sub>O<sub>2</sub>-induced DNA damage and telomere shortening of hu&shy;man fibroblasts derived from different aged individuals whereby it exhibited bi&shy;oprotective effects against free radical attacks (Makpol et al. 2009).</p>\r\n<p>\r\n	Our data showed that the mRNA ex&shy;pression of <em>COLI</em> and <em>COLIII</em> decreased in H<sub>2</sub>O<sub>2</sub>-induced cellular senescence of skin HDFs while the expression of matrix metalloproteinase 1 mRNA (<em>MMP1</em>) was increased when cells aged. Previous study reported among the changes in gene expression in senescent cells, the over expression of matrix metalloprotei&shy;nases (MMPs) was striking resulting in lost of proteins maintaining ultra struc&shy;tural shape (Fisher et al. 2008). Relative overproduction of collagenase in ageing cells was proposed as the matrix-de&shy;grading phenotype of senescent cells (Mawal-Dewan et al. 2002). In addition of that, it has been reported that senes&shy;cent HDFs had decreased expression of sev&shy;eral extracellular matrix components such as collagen I-1&alpha; , collagen III-1&alpha; and elastin besides increased expression of collagenase and stromelysin; two en&shy;zymes that serve to breakdown the extracellular matrix (Fisher et al. 2008).</p>\r\n<p>\r\n	In this study, prolonged <em>C. vulgaris </em>treatment was not able to upre&shy;gulate the mRNA expression of <em>COLI </em>or downre&shy;gulate the mRNA expression of <em>MMP1</em> in H<sub>2</sub>O<sub>2</sub>-induced senescent HDFs even&shy;though the expression of <em>COLIII</em> was in&shy;creased. These findings could be due to the irreversible effects of H<sub>2</sub>O<sub>2 </sub>in skin HDFs. According to studies by Chen et al. (2004) H<sub>2</sub>O<sub>2</sub> induced senescent mor&shy;phological changes and irreversible ar&shy;rest in cell replicative capacity in HDFs F65 cells. With H<sub>2</sub>O<sub>2 </sub>treatment, the cells could not enter the cell cycle again even with mitogen stimulation. The effects of H<sub>2</sub>O<sub>2 </sub>were cumulative and irreversible. This might explain the reason why <em>C. vulgaris</em> could not reverse the effects of H<sub>2</sub>O<sub>2 </sub>in our study. In addition to that, H<sub>2</sub>O<sub>2</sub> was reported to increase the steady-state mRNA levels of collagenase or <em>MMPI</em> in HDFs. It was an important intermediate in the downstream signaling pathway which leads to the induction of increased steady state <em>MMPI</em> mRNA le&shy;vels (Olsen et al. 1989).</p>\r\n<p>\r\n	Compared to other antioxidants, rela&shy;tively few researches had been done to explore the components of <em>C. vulgaris.</em> Therefore the chemical compo&shy;sition of <em>C. vulgaris</em> and its benefi&shy;cial potential espe&shy;cially in preventing skin ageing have not been fully recognized. Various bioactive compounds in <em>C. vulgaris</em> may react to inhibit growth in cells that had been damaged by the oxidative stress induced by H<sub>2</sub>O<sub>2</sub>. <em>C. vulgaris</em> was reported to in&shy;duce apoptosis in order to maintain nor&shy;mal cell homeostasis (Md Saad et al. 2006).</p>\r\n<p>\r\n	In our study, the bioprotective effect of <em>C. vulgaris</em> extractin preventing skin ageing was determined. H<sub>2</sub>O<sub>2 </sub>was addedat passage 6 to induce cellular ageing of the skin while at the same time HDFs were treated with <em>C. vulga&shy;ris </em>extract.Unfortunately there was no extensive study on the influence of H<sub>2</sub>O<sub>2 </sub>towards the effectiveness of prolonged <em>C. vulgaris </em>extract treatment<em>.</em> Therefore for future studies, we would like to suggest that nor&shy;mal replicative senes&shy;cence model shall be used in order to elucidate the protec&shy;tive effects of <em>C. vulgaris</em> extract against skin ageing.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	In skin ageing, <em>COLI</em> and <em>COLIII</em> genes were downregulated while <em>MMPI</em> was upregulated. <em>C. vulgaris </em>extract didmod&shy;ulate the expression of <em>COL</em> and <em>MMP </em>genes by downregulating <em>COLI</em> and upregulating <em>COLIII</em> and <em>MMPI</em> but it did not exert anti-ageing effect.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	This study was funded by Universiti Ke&shy;bangsaan Malaysia grant UKM-GUP-SK-07-21-043.</p>\r\n', 1),
(443, 440, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	<em>Helicobacter pylori </em>is a microaerophilic, gram-negative, slow-growing, spiral-shaped and flagellated bacterium that infects more than 50% of the human population. It resides in gastric-type epi&shy;thelium within the overlying mucous gel and in gastric glands. Invasion of the gastric mucosa by this organism is rarely demonstrated (Chen et al. 1986). It has been identified as the cause of acute and chronic gastritis, peptic ulcer disease (Nomura et al. 1994) and atrophic gastri&shy;tis (Kuipers et al. 1995). The bacterium is also involved in the genesis of gastric adenocarcinoma (Parsonnet et al. 1991) and mucosa-associated lymphoid tissue lymphoma (Wotherspoon 1998). Infection usually occurs early in life and if un&shy;treated persists forever (Blaser 1990). Most people infected with <em>H. pylori</em> es&shy;sentially develop chronic superficial ga&shy;stritis, with only a few patients develop clinical sequelae of infection such as du&shy;odenal and gastric ulceration, gastric adenocarcinoma and gastric lymphoma (Marshall 2002). Difference in the conse&shy;quences of colonization could depend on variation among colonizing <em>H. pylori</em> strains, environmental cofactors or host genetics.</p>\r\n<p>\r\n	Numerous reports from different parts of the world demonstrated that the preva&shy;lence of <em>H. pylori</em> strongly varies between developing and developed countries with high prevalence rates in developing countries (80-90%) and lower rates in developed countries (&lt;40%). Further&shy;more, modes and risk factors of trans&shy;mission, as well as reinfection rates are likely to vary between developing and developed countries (Frenck &amp; Clemens 2003; Rothenbacher &amp; Brenner2003). Within a par&shy;ticular country, marked dif&shy;ferences of <em>H. pylori</em> prevalence have been observed between different ethnic groups. The data suggested that certain ethnic groups are more susceptible to infection with <em>H. pylori</em> than other ethnic groups (Dehesa &amp; Dooley 1991; Goh &amp; Parasakthi 2001). The trend of <em>H. pylori</em> infection in Malaysia has been studied previously (Goh 1997; Goh &amp; Parasakhti 2001; Kaur &amp; Naing 2003). Overall <em>H. pylori</em> prevalence was identified between 13.5 to 55%. However, in these studies, only one method of detection was used, which is serology or histology. <em>H. pylori</em> infec&shy;tion can be determined by various me&shy;thods such as the urea breath test, rapid urease test, histology, culture, se&shy;rology or fecal antigen test. Each method has its pros and cons. Furthermore, pre&shy;vious reports examined <em>H. pylori</em> preva&shy;lence in small numbers of patients with dyspepsia, and recruitment of patients was done in a short period of time. In this study, we attempt to determine the pre&shy;valence of <em>H. pylori</em> infection in a larger numbers of patients with symptoms of dyspepsia using a combination of three methods that detect active <em>H. pylori</em> in&shy;fection.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Patients&rsquo; population</em></p>\r\n<p>\r\n	This is a prospective study on consecu&shy;tive patients who underwent upper ga&shy;strointestinal endoscopy at the Endos&shy;copy Unit, UKMMC from January 1999 to December 2002. Only patients with clear signs of reflux or dyspepsia were in&shy;cluded in the study. Patients who are pregnant, under the age of 10 years old and those with a history of recent anti&shy;biotics use or proton pump inhibitors were excluded from the study. This study has been approved by the UKM Medical Ethics Committee and informed consent was taken from the patients before en&shy;doscopy. Demographic data (age, sex and ethnic group), endoscopic findings and histological diagnoses of all patients were recorded.</p>\r\n<p>\r\n	Patients were classified based on en&shy;doscopic and histological findings into four groups:</p>\r\n<p>\r\n	i)&nbsp;&nbsp;&nbsp;&nbsp; Non-ulcer dyspepsia (NUD); when no disease was found after endoscopy, but presenting with upper abdo&shy;minal pain or epigastric pain, symp&shy;toms re&shy;lated to meals or heartburn, nausea or vomit&shy;ing, gastritis and/or duodenitis.</p>\r\n<p>\r\n	ii)&nbsp;&nbsp;&nbsp; Peptic ulcer disease (PUD); when active peptic ulceration (gastric ulcer and/or duodenal ulcer) was detected after the endoscopic analysis with a mucosal break of at least 0.5 mm in one dimension, with depth or a diag&shy;nosis of peptic ulceration made at a previous endoscopy of upper ga&shy;strointestinal series. Past ulcers also included because <em>H. pylori</em>-associated peptic ulcer disease is regarded as a relapsing and remitting condition that may not be detected at a single en&shy;doscopy.</p>\r\n<p>\r\n	iii)&nbsp;&nbsp; Precancerous and cancerous lesions (PCL); includes intestinal metaplasia, atrophy, dysplasia and gastric carci&shy;noma which is determined by histo&shy;pathological examination.</p>\r\n<p>\r\n	iv)&nbsp; Other diseases; includes gastro-oeso&shy;phageal reflux disease,Barret&rsquo;s oe&shy;sophagus, gastric polyp, oesophageal ulcer and hiatus hernia which is de&shy;tected after endoscopy with no other disease found by histopathologi&shy;cal examination.</p>\r\n<p>\r\n	<em>Biopsy materials and H. pylori culture</em></p>\r\n<p>\r\n	During oesophagogastroduodenoscopy (OGDS), biopsies were taken from an intact antral gastric mucosa using the endos&shy;cope (Olympus, Japan) by gastro&shy;enterologists and surgeons for rapid urease test, histological examination and culture.&nbsp; Biopsy for rapid urease test was inoculated into urea medium and incu&shy;bated aerobically at room tempera&shy;ture, and results were obtained within 24 hours.&nbsp; Biopsy for histopathological ex&shy;amination was fixed in buffered formalin and sent to the Histopathology Unit to look for any pathological changes in&shy;cluding presence of <em>H. pylori</em>.&nbsp; Biopsy for <em>H. pylori</em> culture was placed immediately into a transport medium at 4<sup>o</sup>C and sent to the Microbiology laboratory for culture within six hours after the endoscopy pro&shy;ce&shy;dure.&nbsp; For <em>H. pylori</em> culture, biopsy was inoculated onto Columbia agar contain&shy;ing 7% ox blood, 10 mg/l vancomycin, 5 mg/l trimethoprim, 5 mg/l cefsulodin and 5 mg/l amphotericin B.&nbsp; Plates were in&shy;cubated in a microaerophilic condition obtained by using an anaerobic jar with a gas-generating kit (Campy GasPak, Oxoid) for microaerophilic atmosphere (10% CO<sub>2</sub>, 6% O<sub>2</sub>, 0% H<sub>2</sub>, 84% N<sub>2</sub>) and incubated at 37<sup>o</sup>C for 5 to 7 days. <em>H. py&shy;lori</em> isolates were confirmed by colony morphology, Gram staining and positivity for urease, catalase and oxidase tests.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	Data were analyzed using independent t-test, one way ANOVA and Pearson chi-square, and <em>p</em> value less than 0.05 were considered significant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Study population demography</em></p>\r\n<p>\r\n	A total of 1376 patients were recruited in this study. Of these, 670 were male and 706 were female, with age ranges from 13 to 99 years (mean age 52.6, standard deviation 15.5). Patients were from three major ethnic groups comprising 541 Ma&shy;lays, 630 Chinese and 205 Indians. The distribution of mean age, gender and ga&shy;stroduodenal diseases in the study pop&shy;ulation are shown in Table 1. Data showed that the mean age was higher in patients with severe diseases such as peptic ulcer disease and precancerous and cancerous lesions compared to pa&shy;tients with non-ulcer dyspepsia. More male patients had peptic ulcer compared to female patients, whereas non-ulcer dyspepsia were high in female patients.</p>\r\n<p>\r\n	Table 2 showed the distribution of dis&shy;ease groups among patients of three different ethnic groups.&nbsp; Patients with non-ulcer dyspepsia were high in Malays and Indians compared to Chinese. Pep&shy;tic ulcer disease and precancerous and cancerous lesions were high in Chinese patients compared to Malays and In&shy;dians.</p>\r\n<p>\r\n	<em>H. pylori infection</em></p>\r\n<p>\r\n	Specimens were considered to be <em>H. pylori</em> positive if either rapid urease test or culture or histopathological examina&shy;tion methods gave positive results. Of 1376 patients, 424 (30.8%) were positive for <em>H. pylori</em>. Ninety nine were Malays, 232 were Chinese and 93 were Indians.&nbsp; Of these, 225 patients were male and 199 were female. The mean age was 54 (age range: 15 &ndash; 98) with standard devi&shy;ation of 14.7. Table 3 shows the charac&shy;teris&shy;tics of patients with and without <em>H. pylori </em>infection. There was a significant differ&shy;ence in mean age between <em>H. py&shy;lori</em>-positive and <em>H. pylori</em>-negative pa&shy;tients (<em>p</em> = 0.027). <em>H. pylori</em>-positive infec&shy;tion was more prevalent among males compared to females (<em>p</em> = 0.03). There was a sig&shy;nificant difference in <em>H. pylori</em> infection between the different ethnic groups, In&shy;dians had the highest infection rate of 45.4% (93/205), followed by Chi&shy;nese with 36.8% (232/630) and Malays 18.3% (99/541). There was also a signifi&shy;cant difference between <em>H. pylori</em> infec&shy;tion and severity of gastroduodenal dis&shy;eases. <em>H. pylori</em>-positive infection was high in patients with severe gastroduo&shy;denal dis&shy;eases (peptic ulcer disease and precan&shy;cerous and cancerous lesions) compared to patients with non-ulcer dys&shy;pepsia and other disease groups.</p>\r\n<div>\r\n	<p>\r\n		Table 4 showed the characteristics of <em>H. pylori</em>-positive patients from the differ&shy;ent ethnic groups. Chinese patients at older age were more prevalent to be in&shy;fected compared to Indians and Malays. In all ethnic groups, <em>H. pylori</em> prevalence was similar in males and females. Sig&shy;nificant difference was seen between disease groups and patients&rsquo; ethnicity. In Malays and Indians, the prevalence rate of non-ulcer dyspepsia was high com&shy;pared to Chinese, whereas peptic ulcer disease and precancerous and cancer&shy;ous lesions were seen more in Chinese patients compared to Malays and In&shy;dians.</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		Gastric ulcer, duodenal ulcer and gastric cancer are common and serious gastro&shy;duodenal disease but occur in only a mi&shy;nority of people. Non-ulcer dyspepsia is one of the most frequently encountered disorders in gastroenterology clinics worldwide (Talley et al. 1992). Previous studies showed that the cumulative risk of ulcer development in non-ulcer dys&shy;pepsia patients ranged from 1% to 21% during a follow up period of 1 &ndash; 10 years (Hsu et al. 2002). However, the risk fac&shy;tors influencing the subsequent devel&shy;opment of peptic ulcer in non-ulcer dys&shy;pepsia patients remain unclear. In the present study, the non-ulcer dyspepsia group constituted the largest of patients (69%), whereas 19.2% patients had pep&shy;tic ulcer disease. A small proportion of patients (8.3%) had precancerous le&shy;sions and 0.5% of patients were diag&shy;nosed with gastric carcinoma. Patients were from a single center i.e., from the Endoscopy Unit of UKMMC which is a tertiary and referral center for the coun&shy;try. This offers uniformity in standards for endoscopy, definitions of dyspepsia and histology.</p>\r\n	<p>\r\n		The important variables for increased prevalence of severe gastroduodenal diseases were age and male patients. The mean age of patients was higher in those with severe gastroduodenal dis&shy;eases and more male patients had gas&shy;tric or duodenal ulcer. This could be ex&shy;plained by the presence of age related changes in the gastric mucosal defense in the elderly (Hsu et al. 2002). A study in humans (Cryer et al. 1992) demonstrated that gastric mucosal prostaglandin con&shy;tent declines with age. Feldman and Cryer (1998) also showed that advanced age is associated with a significant de&shy;cline in gastric bicarbonate, sodium ions and non-parietal fluid secretion. Thus aging is associated with selective as well as specific changes in the gastric mu&shy;cosal defenses that may predispose to the development of severe disease.</p>\r\n	<p>\r\n		The study population comprised pa&shy;tients with symptoms of dyspepsia from three major ethnic groups; Malays, Chi&shy;nese and Indians. Chinese formed the largest group of patients, followed by Malays and Indians, which is consistent with the high prevalence of gastroduo&shy;denal diseases in Chinese compared to other ethnics. More Chinese patients were diagnosed with severe disease compared to other ethnics and the pat&shy;tern has not change so much since 1994 (Haron et al. 1994; NCR 2003).</p>\r\n	<p>\r\n		In this study, <em>H. pylori</em> infection status was determined by rapid urease test, culture or histological assessment on gastric biopsy. Urease test has been rendered a highly sensitive (90 &ndash; 93%) and specific (93 &ndash; 95%) diagnostic test (Laine et al. 1996). The test is fast and inexpensive for detecting <em>H. pylori</em> infec&shy;tion. Culture is the gold standard for identifying bacterial infection and culture of <em>H. pylori</em> provide the <em>H. pylori</em> isolate for further characterization. Histological examination does allow for definitive di&shy;agnosis of infection, the severity of ga&shy;stritis as well as the presence of intes&shy;tinal metaplasia, atrophy, dysplasia and gastric carcinoma to be assessed at the same time. In this study, an overall pre&shy;valence of <em>H. pylori</em>-infected patients with symptoms of dyspepsia was 30.8% and increased <em>H. pylori</em>-positive infection rates were observed with the increased severity of disease. Gender and age did prove to be significant risk factors for <em>H. pylori</em> infection in this study, and it is con&shy;sistent with other studies which showed these variables to be independent risk factors as well (Goh 1997). The ethnic composition in both studies was almost similar in which the majority of patients were Chi&shy;nese. In contrast, a study in North Eastern Peninsular Malaysia showed that gender and age were not significant risk factors for <em>H. pylori</em> infec&shy;tion. In the study, Ma&shy;lays constituted the majority of the study population with 64%, whereas Chinese were 28% and Indians were 5.2% (Kaur &amp; Naing 2003). These results showed that the composi&shy;tion of the ethnic groups play a role in determining the risk factors for <em>H. pylori</em> infection.</p>\r\n	<p>\r\n		The composition of ethnic groups re&shy;cruited may also affect the overall pre&shy;valence rates of <em>H. pylori</em> infection. In the present study, the population comprised of 39.3% Malays, 45.8% Chinese and 14.9% Indians. Whereas, in the study which comprised of 17% Malays, 53% Chinese and 30% Indians, results showed more than 45% of <em>H. pylori</em> pre&shy;valence (Goh 1997). In the study in which <em>H. pylori</em> pre&shy;valence rates was less than 15%, the major study population com&shy;prised of Malays (64%), fol&shy;lowed by Chinese (28%) and 5.2% Indians (Kaur &amp; Naing 2003).</p>\r\n	<p>\r\n		The differences in the prevalence of <em>H. pylori</em> between the ethnic groupswere consistent with the findings by other en&shy;doscopy-based and seroepidemiological studies (Goh 1997; Goh &amp; Parasakthi 2001; Kaur &amp; Naing 2003) previously re&shy;ported in the region. Further observations revealed differences in the distribution of non-ulcer dyspepsia, peptic ulcer disease and pre&shy;cancerous and cancerous lesions in <em>H. pylori</em>-positive patients among vari&shy;ous ethnic groups. Chinese shows con&shy;sistently higher rates of severe disease compared to other ethnics. Although In&shy;dians have a higher prevalence of <em>H. pylori</em> infection, a lower frequency of peptic ulcer disease was observed com&shy;pared to other ethnic groups. The differ&shy;ence in the prevalence of <em>H. pylori</em> and gastro&shy;duodenal diseases among patients of different ethnic groups living in the same country is interesting, and may provide valuable insights into the possi&shy;ble mode of pathogenesis of the infec&shy;tion. Patho&shy;genic <em>H. pylori</em> strains, host genetics and environmental factors may account for the differences. This needs to be investi&shy;gated.</p>\r\n	<p>\r\n		There were some limitations in this study which should be taken into ac&shy;count. It is almost impossible to be com&shy;pletely cer&shy;tain of the clinical diagnosis. A definite diagnosis of non-ulcer is difficult. Patients with only gastritis at endoscopy may de&shy;velop ulcer disease later in life and therefore may have been misclassi&shy;fied in the present study. Furthermore, our limi&shy;tation is that different gastroen&shy;terologists or surgeons did the endos&shy;copic observa&shy;tion on each patient, and different pa&shy;thologists analyzed the biopsy speci&shy;mens, although they used the same scale or scoring system. This may affect the clinical diagnosis of the patients and classification of our patients&rsquo; disease group. Another important limitation in our study is that the number of patients in each disease group is not equal between different groups since peptic ulcer and precancerous and cancerous lesions constitute a small proportion of patients with dyspepsia.</p>\r\n	<p>\r\n		In conclusion, results of this study showed that there was a significant dif&shy;ference in <em>H. pylori</em> infection among eth&shy;nic groups in which, Indians had the highest infection rate followed by Chi&shy;nese and the lowest in Malays. <em>H. pylori</em> infection rates were high in patients with severe gastroduodenal diseases. How&shy;ever, the distribution of severe gastrodu&shy;dodenal diseases does not reflect the prevalence of <em>H. pylori</em> infection among different ethnic groups. The reasons for racial differences in <em>H. pylori</em> infection and distribution of severe gastroduodenal diseases cannot be explained. This may be related to the host or bacteria genetic factors, and sociocultural beha&shy;vior of the particular ethnic and this needs further investigation.</p>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(444, 441, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Monitoring of residual disease (MRD) in leukaemic patients offers a means of evaluating the efficacy of the treatment given. In childhood acute lymphoblastic leukaemia (ALL), relapses are reported in at least 20% of children who are treated with contemporary programs of chemotherapy. However, monitoring MRD in childhood ALL may create the possibility of tailoring treatment according to patient&rsquo;s response, where a more in&shy;tensive therapy could be given to pa&shy;tients with high levels of MRD and a less intense one to those without MRD. Vari&shy;ous methods of detection of MRD in childhood precursor B-ALL, such as by morphology, karyotyping, in situ hybridi&shy;zation, immunofluorescence, cell culture, Southern blotting and polymerase chain reaction (PCR) analysis have been de&shy;scribed. However, the evaluation of resi&shy;dual disease in acute leukaemia is most often carried out by morphological analy&shy;sis of bone marrow aspirates with the criterion of free from residual disease (complete remission) denotes fewer&nbsp; than 5 % of blasts in the bone marrow cell population. Unfortunately, this method does not allow identification of low levels of the disease and patients in morpho&shy;logical remission may still have up to 10<sup>10</sup> leukaemia cells (Campana &amp; Pui 1995; Campana 2003).&nbsp;</p>\r\n<p>\r\n	Sensitive techniques to detect MRD may allow better estimation of the leu&shy;kaemia burden, provides information on residual disease and aids in therapeutic strategies. Flow cytometric detection of MRD is based on the identification of immunophenotypic combination markers expressed on leukaemic cells but not on normal haemopoietic cells. It is based on the identification of uncommon pheno&shy;types or aberrant markers that are found at diagnosis. At certain points of time during and after chemotherapy, the same phenotypes or aberrant markers are analyzed and quantified. If expression of these markers is less than 1%, the pa&shy;tient is considered free from MRD. How&shy;ever, for the detection of a small number of leukaemic cells, a large number (10<sup>5 </sup>- 10<sup>6</sup>) of cells is needed for screening. Thus flow cytometry can be used as a tool to predict relapse of leukaemia pa&shy;tients particularly when subsequent anal&shy;ysis of marrow MRD levels shows an in&shy;creasing trend.</p>\r\n<p>\r\n	The close relationship between the size of tumour burden and the curability of acute leukaemia is well established. In ALL, a high white blood cell count (WBC) at diagnosis is indicative of a large tu&shy;mour mass and it is a poor prognostic indicator (Campana 2003, Pui &amp; Crist 1994). Other presenting features such as age, sex, lymphadenopathy, organome&shy;galy and the immunophenotype of blast cells are also known to have prognostic values in determination of re&shy;mission in&shy;duction rate, remission dura&shy;tion and long-term survival of patients. Other pa&shy;rameters such as haemoglobin and platelet count may play some role; how&shy;ever, myeloid associated markers have been proven to have no adverse prog&shy;nostic significance in children (Pui et al. 2008). These parameters have been used to classify patients into standard or high-risk groups before appropriate che&shy;motherapy is started. From previous stu&shy;dies, patients in the high-risk group that received standard risk of chemotherapy regime have a higher chance of relapse.</p>\r\n<p>\r\n	The aim of this study was to establish the detection of residual disease by im&shy;munophenotyping using flow cytometry in childhood ALL during their induction and continuing chemotherapy.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This descriptive cross sectional study was conducted in children with precursor B-ALL aged between one and 12 years, diagnosed in UKM Medical Centre (UKMMC) from June 2001 to October 2003. It was carried out under a protocol approved by the Institutional Review and Ethics Committee. We obtained bone marrow samples from patients with in&shy;formed consent at diagnosis, and at vari&shy;ous time points that have been defined for standardized analysis of chemothe&shy;rapy treatment based on the UKALL 97 protocol (Working Party On Leukaemia In Children, Revised November 1999). The time points of analysis were: following induction chemotherapy (day-28) and during the continuation therapy (week-12 and week-20) for MRD as&shy;sessment. The specimens of the patients were processed in the Haematology Unit, De&shy;partment of Pathology UKMMC. All pa&shy;tients were followed-up for five years to ascertain their survival status.</p>\r\n<p>\r\n	Bone marrow smears were made from the marrow aspirations of the patients for morphological analysis and the remain&shy;ing of the marrow specimens were col&shy;lected in ste&shy;rile ethylenediamine tetra-acetic acid (EDTA) tubes for immuno&shy;phenotyping analysis. Differential counts of the pa&shy;tients from peripheral blood were also obtained at the same time. The marrow smears were stained for May-Grunwald-Giemsa&rsquo;s (MGG) and perox&shy;idase cyto&shy;chemistry. The morphological assess&shy;ments of bone marrow smears were ob&shy;served by two Pathologists and the per&shy;centage of lymphoblasts were recorded. Mononuclear cells from bone marrow aspirates collected in EDTA, were sepa&shy;rated on a density gradient using lym&shy;phoprep (HISTOPAQUE-1077), immu&shy;nolabelled with a panel of mono</p>\r\n<p>\r\n	clonal antibodies conjugated with FITC and/or PE and analyzed by FACScan flowcyto&shy;meter (Becton Dickinson) for immunophe&shy;notyping. Analyses of data were done using Cell Quest software (Becton Dickison).</p>\r\n<p>\r\n	Diagnosis of precursor B-ALL was made based on the presence of surface expression of CD19 and cytoplasmic ex&shy;pression of CD 22 with negative cytop&shy;lasmic myeloperoxidase (MPO). The acute lymphoblastic leukaemia cases were included in the study if the blast cells expressed at least one of the fol&shy;lowing combined immunomarkers:</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; i.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; CD19/CD34</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; ii.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; CD33/CD19</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; iii.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; CD13/CD19</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; iv.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; CD33/CD10</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; v.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; TdT</p>\r\n<p>\r\n	For MRD detection, bone marrow sam&shy;ples from all patients during follow-up visits to the hospital were analyzed with the same immunomarkers that were present at diagnosis. Figures 1 and 2 illustrate the examples of cases that were MRD positive and MRD negative respec&shy;tively. Morphological assessments were also compared with the flow cytometry immunophenotyping results. Bone mar&shy;row samples from pediatric cases of idi&shy;opathic thrombocytopenic purpura (ITP) and non-haematological malignancy without bone marrow involvement were used as controls.</p>\r\n<p>\r\n	Demographic and clinical data such as age, sex, race, presence of lymphadeno&shy;pathy, hepatomegaly or splenomegaly and blood profile of the patients upon diagnosis and at day-28 post induction chemotherapy, were also recorded.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	<em>Sample population</em></p>\r\n<p>\r\n	From June 2001 to October 2003, 49 children aged one to 12 years with pre&shy;cursor B-ALL were diagnosed in UKMMC. However, minimal residual dis&shy;ease (MRD) could only be studied in only 38 cases. Eleven children had to be ex&shy;cluded from the study because their bone marrow samples were not available at post chemotherapy or there were no suitable MRD markers detected at diag&shy;nosis. Table 1 shows the ranges of age, sex and race of patients in this study.</p>\r\n<p>\r\n	<em>Clinical findings at diagnosis</em></p>\r\n<p>\r\n	Most of the children with precursor B-ALL presented with lymphadenopathy (76.3%), hepatomegaly (89.5%) and splenomegaly (76.3%). Some of these cases had very large liver and spleen (&ge;5.0 cm and below subcostal mar&shy;gin) i.e. 11 (28%) and 8 cases (21%) re&shy;spec&shy;tively.</p>\r\n<p>\r\n	<em>White blood cells at diagnosis</em></p>\r\n<p>\r\n	Table 2 illustrates the haematological characteristics of the patients at diagno&shy;sis. Most of the cases presented with white blood cell counts (WBC) of less than 50.0x10<sup>9</sup>/L. Only four cases (10.5%) had WBC more than 50.0x10<sup>9</sup>/L with the</p>\r\n<p>\r\n	highest count being 322.0x10<sup>9</sup>/L (Table 2) and the lowest WBC was 2.0x10<sup>9</sup>/L. The mean WBC was 29.07x10<sup>9</sup>/L.&nbsp; Most of the patients showed significant reduc&shy;tion in the WBC at day-28 post chemo&shy;therapy.</p>\r\n<p>\r\n	<em>Residual leukaemic cells by flow cytometry (Table 3)</em></p>\r\n<p>\r\n	Residual leukaemic cells (MRD) had been analyzed by flow cytometry at the specified points of time as mentioned earlier. However, only 30 cases at day-28 of chemotherapy were available for MRD analysis, where 11 cases were found to be positive for MRD and 19 cases had no residual disease.&nbsp; At week-12 of therapy, two cases had to be ex&shy;cluded due to inadequate marrow sam&shy;ples for MRD analysis but the number of cases that became negative for MRD had increased to 25 patients (69.0 %).&nbsp;</p>\r\n<p>\r\n	However, at week-20, fewer samples were available for the analysis. There were inadequate specimens in ten pa&shy;tients, one patient had defaulted the fol&shy;low-up treatment and sought traditional medicine while another had succumbed to sepsis.</p>\r\n<p>\r\n	<em>Correlation between demographic, clinical and laboratory data of ALL cases at diagnosis and the residual disease (MRD) at day-28 of therapy</em></p>\r\n<p>\r\n	There were only 30 cases for MRD as&shy;sessment at day-28 of chemotherapy (Table 4). There was no correlation be&shy;tween sex, race or age of the patients at diagnosis with MRD findings at day-28 (p&gt;0.05). There was no correlation be&shy;tween lymphadenopathy and hepatos&shy;plenomegaly at diagnosis with MRD at day-28 (p&gt;0.05). Correlation between WBC, haemoglobin (Hb) and platelet counts at diagnosis with MRD at day-28 were also not significant (p&gt;0.05). Cases with a haemoglobin count of less than 8g/dl and WBC of less than 50.0 x 109/L were negative for MRD. There was also no correlation between the immuno&shy;phe&shy;notype of the blast cells and pres&shy;ence of myeloid aberrant markers&nbsp; at di&shy;agnosis with MRD at day-28 (p&gt;0.05).</p>\r\n<p>\r\n	<em>Correlation between morphological and flowcytometric assessments of residual disease (MRD) of ALL cases</em></p>\r\n<p>\r\n	In this study, 35 cases achieved com&shy;plete marrow remission at day-28 of in&shy;duction chemotherapy by morphological examination. One case (Table 6; case 6) had a borderline blast cells percentage of 5%. Analyses for MRD by flow cytome&shy;try at day-28 could be performed in only 30 cases and 11 cases were found to be positive for MRD (Table 5) but the mor&shy;phological examination of their marrow specimens showed blast counts of less than 5%.</p>\r\n<p>\r\n	For the week-12 analysis, bone marrow aspirations could be performed only on 36 of 38 cases and the correlation be&shy;tween marrow findings and MRD at week-12 was found to be statistically sig&shy;nificant (p&lt;0.05). Flow cytometric analy&shy;sis at week-12 showed that there were nine cases with positive MRD although their morphological marrow assessments showed that the percentages of the blast cells were less than 5% (Table 5). One of the cases with positive MRD (case 6) had also not achieved complete marrow remission at day-28 (Table 6). Two cases were found to have blast cells of more than 5% of total nucleated cells by mor&shy;phological examination and they were also positive for MRD by flow cytometric analysis.</p>\r\n<p>\r\n	<em>Sequential determination of residual disease and survival </em></p>\r\n<p>\r\n	There were only 3 cases (Case 1, 3 and 6) with persistent MRD at week-20 of chemotherapy. These cases however, had no MRD assessment at day-28 (Ta&shy;ble 6). Case 1 and 3 had marrow remis&shy;sion morphologically at day-28 but case 6 had borderline blast cell percentage (5%). Case 1 relapsed after one year while case 3 after six months of treat&shy;ment. Both succumbed to treatment complications.&nbsp; To date, case 6 is still alive, which is more than 5 years after diagnosis.</p>\r\n<p>\r\n	There were four cases with persistent MRD from day-28 until week-12 of ther&shy;apy but at week-20, three cases achieved a negative MRD status. How&shy;ever, MRD analysis could not be done on one case (case 38) due to insufficient sample. To date, only one patient (case 5) had passed away after 3 years of di&shy;agnosis.</p>\r\n<p>\r\n	There were 18 cases that were always negative for MRD. However, eight of them died within 12 weeks to 4 years af&shy;ter di&shy;agnosis. Case 8 had relapsed after 3 years of treatment; case 39 had de&shy;faulted and relapsed while others re&shy;mained in remission but unfortunately had succumbed to sepsis. Twenty four patients were still alive after 5 years of follow up.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Acute lymphoblastic leukaemia (ALL) is the most common malignant disease-af&shy;fecting children and in Malaysia approx&shy;imately 75% of ALL cases are younger than 20 years of age (Lim et al. 2002). Most of the cases in this study were in the good prognostic age group i.e. two to ten years of age (73.7%) but ma&shy;jority of the cases were males (81%). A longer survival of cases in age group between two and ten years have been reported, which was more than one-and-a-half times as compared to patients&nbsp; with age of less than two years or more than 10 years, when&nbsp; treated in an iden&shy;tical man&shy;ner (Pui et al. 2008).</p>\r\n<p>\r\n	Gender has little or no impact on the success of remission induction and the relapse frequency during the first month of treatment. However, after discontinua</p>\r\n<p>\r\n	tion of therapy, boys continue to expe&shy;rience a higher incidence of relapse due to relapse in the testes as well as in the bone marrow (Chessells et al. 1995). In this study, 12 of 31 (38.7%) male pa&shy;tients had relapsed and died within five years. The number of female patients involved in this study was small for a comparative study.</p>\r\n<p>\r\n	Ethnically, the majority (78.9%) of the cases were Malays, followed by Chinese (13.2%) and Indians (7.9%). This obser&shy;vation is consistent with the data of the National Cancer Registry published in 2002, where Malays have the highest incidence of leukaemias (Lim et al. 2002).</p>\r\n<p>\r\n	The sizes of peripheral lymph nodes, liver and spleen provide an indirect mea&shy;surement of leukaemic cell burden. Sev&shy;eral studies have demonstrated that massive lymphadenopathy; hepatome&shy;galy and splenomegaly adversely affect remission duration and survival (Ojala et al. 1995). However, mild to mod&shy;erate in&shy;creases in the size of lymph nodes and abdominal organs do not ap&shy;pear to influ&shy;ence prognosis of ALL cases. In this study, most of the cases pre&shy;sented with lymphadenopathy (76.3%), hepatome&shy;galy (89.5%) and splenome&shy;galy (76.3%), and enlarged liver and spleen of 5.0 cm or more were seen in 11 cases (28%) and 8 cases (21%) re&shy;spectively.&nbsp;</p>\r\n<p>\r\n	The total white blood cell count (WBC) at the time of diagnosis is the single most powerful determinant of remission induc&shy;tion, remission duration, and long-term survival for all age groups (Pui et al. 2008). Patients with higher WBC at diag&shy;nosis have a higher risk for treatment failure than patients with lower WBC, and often have bulky extramedullary disease. WBC of more than 50.0 x 10<sup>9</sup>/L is an in&shy;dication of poor prognosis (Pui et al. 1998; Shu &amp; Chen 2005).</p>\r\n<p>\r\n	In this study, the clinical and haemato&shy;logical characteristics of children with ALL at diagnosis were compared with the residual disease status detected by flow cytometry, to assess for any correlation. MRD detection on completion of induc&shy;tion therapy was not significantly related to age, gender, race, lymphadenopathy and hepatosplenomegaly, leukocyte count, haemoglobin level, platelet count, immunophenotype of lymphoblast, and presence or absence of myeloid aberrant markers. It appears that the above fac&shy;tors could not be used to predict MRD status at day-28.</p>\r\n<p>\r\n	Early clearance of leukaemic cells is a favourable prognostic indicator in child&shy;hood ALL. However, identification of re&shy;sidual leukaemic cells by light micro&shy;scopy is subjective and lacks sensitivity. Common therapeutic protocols have considered 5% of blast cells in the bone marrow as the morphologic limit for con&shy;sidering complete remission of ALL pa&shy;tients. Even though intensification of chemotherapy, risk stratification and supportive care have improved the out&shy;come of childhood ALL, 20% of children still succumb to relapse. Thus, a patient declared to be in complete clinical remis&shy;sion might, harbour as many as 10<sup>10</sup> leu&shy;kaemic cells. The study of MRD has im&shy;proved the estimation of the number of blast cells present in the bone marrow during complete remission and during or after therapy. Thus it may be helpful to design a risk-adapted therapy in patients with acute leukaemia. The ability to detect residual leukaemic cells while they are still sensitive to treatment would al&shy;low for potentially curative intervention. The persistence of small numbers of leu&shy;kaemic cells post induction chemothe&shy;rapy in childhood ALL, indicates the re&shy;quirement of prolonged therapy (2.5 to 3 years) to reduce the relapse hazard (Pui &amp; Crist 1994; Hoelzer 1994).</p>\r\n<p>\r\n	The sensitivity of MRD detection using flow cytometry depends on the numbers of cells analyzed and also on the MRD markers used. For optimal flow cytometry results it is imperative that the bone mar&shy;row specimen contain a large number of viable with a good separation of mono&shy;nuclear cells. The sample preparation and live gate acquisition should be per&shy;formed within 24 hours. If the samples have remained unprocessed for a longer duration, there will be changes in the FSC/SSC pattern and the increasing population of dying or apoptotic cells will interfere with the analysis and interpreta&shy;tion of the results. Post chemotherapy bone marrow samples also have a large number of new regenerating normal lym&shy;phoid cells (haematogones) which may express CD34, CD19, CD10 and TdT and may affect the analysis of results (Coustan-Smith et al. 2006).</p>\r\n<p>\r\n	We had used a two-color flow-cytome&shy;try for measurement of MRD. Patients with a high tumour burden at diagnosis and have MRD more than 1% post che&shy;motherapy are more likely to relapse early (Coustan-Smith et al. 2000). In this study, there were three cases with per&shy;sistent MRD positivity and the correlation study between marrow findings and MRD status at week-12 was found to be statis&shy;tically significant (p&lt;0.05). Two of these patients had an early relapse and suc&shy;cumbed to the disease. Marrow remis&shy;sion status post chemotherapy of pa&shy;tients with MRD less than 1% were questionable (Coustan-Smith et al. 2000). There were 18 cases in this study that had not shown MRD positivity at any point of time but eight of them died within a period of 12 weeks to 4 years after di&shy;agnosis.</p>\r\n<p>\r\n	Coustan-Smith et al. (2006) showed that by using multi-coloured flow-cyto&shy;metry, MRD can be detected up to 0.01% and the cases that showed less than 0.01% MRD positivity at the end of re&shy;mission induction are likely to have an excellent treatment outcome, compared to those with more than 0.01% (Coustan-Smith et al. 2006).</p>\r\n<p>\r\n	In the Paediatric Haemato-Oncology Unit UKMMC, stratification of intensive treat&shy;ment usually depends on the pre&shy;senting WBC and age of the patient. Following induction chemotherapy, pa&shy;tients who had more than 5% of blast cells in the bone marrow morphologically and pa&shy;tients with nervous system (CNS) or tes&shy;ticular relapse will be given a more inten&shy;sive chemotherapy regimen. Thus MRD analysis by flow cytometry can also be used to design a risk-adapted therapy in childhood ALL. The ability to detect resi&shy;dual leukaemic cells while they are still sensitive to treatment would allow more time, and potentially curative inter&shy;ven&shy;tion. The persistence of small num&shy;bers of leukaemic cells post induction chemothe&shy;rapy in childhood ALL indicate the re&shy;quirement of prolonged therapy (2.5 to 3 years) to reduce the relapse hazard (Campana 2003; Hoelzer 1994).</p>\r\n<p>\r\n	This study was unable to prove that the risk of relapse was high in patients with persistent MRD since a few cases which were MRD negative also relapsed within 4 years after diagnosis.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	This study showed that there was no sig&shy;nificant correlation between MRD positiv&shy;ity by flow cytometry with the demo&shy;graphic, clinical or haematological data at diagnosis. However, there was a signifi&shy;cant correlation between MRD positivity by flow cytometry with morphological analysis of blast cells by light micro&shy;scopy. For better evaluation of MRD, a three or four&nbsp; colour flow cytometry anal&shy;ysis should be used and a larger cohort of ALL cases are required, and would then aid in providing a basis for future clinical decision making in the manage&shy;ment of ALL cases.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	The authors gratefully acknowledge and thank Mrs Sivagengei for her technical assistance and advice regarding flow cytometry.</p>\r\n', 1),
(445, 442, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	The World Health Organization has de&shy;clared that the number of people with di&shy;abetes mellitus (DM) is rapidly in&shy;creasing and has become of major public health con&shy;cern (King &amp; Rewers 1991; WHO 1992). The number of diabetic patients in develop&shy;ing countries is expected to in&shy;crease by more than two-folds from 84 million in 1995 to 225 million in 2025 (Aboderin et al. 2002). In Malaysia, the prevalence of adult diabet&shy;ics has doubled over a 10-year period with the prevalence in 2006 reported as 14.9%, the highest among the Asian countries (NHMSIII 2006).</p>\r\n<p>\r\n	Complications affecting the lower limbs are one of the most common manifesta&shy;tions of diabetes. Approximately 15% of those with diabetes will eventually suffer from foot ulceration during their lifetime (WHO 1992). It is well recognized that diabetic patients experience decreased sensation of their feet because of peri&shy;pheral neuropathy which is the result of a complex interplay between a number of factors such as limited joint mobility (LJM), altered foot pressures and level of gly&shy;caemic control (Boulton et al. 1983; Caputo et al. 1994; Reiber et al. 1999).The loss of sensation caused by poorly controlled diabetes is also thought to re&shy;duce the protective sensation of the foot. The repetitive trauma to the plantar areas increases the risks of foot ulceration, which may lead to foot amputation in se&shy;vere uncontrolled cases.</p>\r\n<p>\r\n	Factors such as age, gender, diabetic control, presence of vascular disease, and extrinsic sources of trauma were theoreti&shy;cally reported to be associated with the development of peripheral neu&shy;ropathy amongst diabetic patients (Adler et al. 1997; Tesfaye et al. 1996). To date, there is limited evidence regarding the risk fac&shy;tors for the development of peri&shy;pheral neuropathy amongst diabetic pa&shy;tients. Hence, this study aimed to identify risk factors associated with the develop&shy;ment of peripheral neuropathy in DM pa&shy;tients and their association with the se&shy;verity of peripheral neuropathy amongst these pa&shy;tients. The results from this study will be beneficial for the health care providers to initiate early preventive measures to the patients.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Design</em></p>\r\n<p>\r\n	This was a cross-sectional study con&shy;ducted amongst patients diagnosed with DM attending outpatient clinics in UKM Medical Centre, Kuala Lumpur. The study was conducted from January to August 2008, involving diabetic patients in three out-patient clinics i.e. primary care centre, medical endocrine clinic and orthopaedic clinic.</p>\r\n<p>\r\n	<em>Subjects </em></p>\r\n<p>\r\n	All diabetic patients aged 18 to 70 years old attending the clinics were in&shy;vited to participate in the study. Written consent was obtained before patients were sub&shy;jected to assessments for the study. The diagnosis of DM was estab&shy;lished from re&shy;views of the patients&rsquo; medi&shy;cal records. Consented patients were given a checklist documenting duration of diabetes, medical co-morbidities, smok&shy;ing habits and pres&shy;ence of foot ulcera&shy;tion. Patients were then examined for the presence of clinical peri&shy;pheral neuropa&shy;thy. Exclusion criteria in&shy;cluded patients with limb amputation, gross foot defor&shy;mity (e.g: Charcot joints, hallux valgus), and existing peripheral neuropathy in patients.&nbsp; Controls were recruited from non-diabetes mellitus (NDM) subjects that were able to walk normally, had no history of foot problem and attending the clinic as the subjects&rsquo; accompaniment during the study period.</p>\r\n<p>\r\n	<em>Study instrument</em></p>\r\n<p>\r\n	Neuropathy screening was performed us&shy;ing the Semmes-Weinstein monofila&shy;ment (SWMF) 5.07/10g that was used as a quantitative assessment of the cutane&shy;ous pressure perception. With both eyes closed, the patient informed the investi&shy;gator when he or she could feel the fila&shy;ment applied to a non-callus site at 8 plan&shy;tar locations. Neuropathy was con&shy;firmed when more than half&nbsp;&nbsp; responses were in&shy;correct (Perkins et al. 2001). The Neuro&shy;pathy Disability Score (NDS) was used to quantify the severity of the neu&shy;ropathy.&nbsp; The Achilles tendon reflexes were ex&shy;amined: a score of zero (0) was given if the reflexes were normal, one (1) if the reflexes could be elicited with rein&shy;forcement and two (2) if the reflexes were absent.&nbsp; Sen&shy;sory testing included pin prick using neuro&shy;tip, vibration using a biothesiometer with a cut-off value of 25 Volts to determine vibra&shy;tion perception and temperature testing using a cold tuning fork at the great toe: a score of zero (0) was given for each normal result and one (1) for each abnormal result. The summation of scores of each modal&shy;ity was entered as the NDS. A score of 1 and 2 was considered normal, whereas score of 3 to 5 indicated mild peripheral neuro&shy;pathy, 6 to 8 indicated moderate peripheral neuropathy and 9 to 10 indi&shy;cated severe neuropathy. Finally, the glycosylated hae&shy;moglobin (HbA1c) within the past six months was used to ascer&shy;tain the level of glycaemic control of the patient.</p>\r\n<p>\r\n	<em>Statistical analysis</em></p>\r\n<p>\r\n	Statistical analysis was conducted using the SPSS version 12.0. Descriptive analy&shy;sis was used for categorical data and analysis of mean, median and variance. The correlation of continuous variables and NDS were analyzed using Spear&shy;man&rsquo;s Rank order. For the correlation of categorical variables and NDS, Mann-Whitney U test was used to determine the difference. Logistic regression analysis was applied to look for risk factors asso&shy;ciated with peripheral neuropathy. A p value of &lt;0.05 was considered significant.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total of 91 subjects (72 DM patients; 19 NDM volunteers) were recruited. The demographic characteristics are shown in Table 1. For the DM group, 37.5% had DM for more than 15 years, 20.8% be&shy;tween 10 to 15 years, 20.9% between 5 to 10 years while the remaining 20.8% less than five years.There were 49 (41.7%) males.</p>\r\n<p>\r\n	Among the DM patients, 79.1% demon&shy;strated various levels of peripheral neu&shy;ropathy (Table 1). Subjects with callus or ulcers had higher NDS than those with&shy;out (Z: -2.36, p: 0.003). There was a posi&shy;tive correlation between the age of di&shy;abetic subjects and the NDS score (r: 0.36, N: 72, p: 0.02), with older DM sub&shy;jects showing higher NDS. Weight of the DM subjects also demonstrated a posi&shy;tive correlation to the NDS (r: 0.35, N: 72, p: 0.03), with heavier diabetic subjects showing higher NDS. Although NDS seemed to have significant correlation with HbA1c (p: 0.005) and duration of di&shy;abetes (p&lt;0.005), it was not signifi&shy;cant in the logistic regression analysis. In con&shy;trast, NDS was not found to have an as&shy;sociation with gender (<em>P</em>=0.179), ever smoked (<em>P</em>=0.351) and history of ulcer (<em>P</em>=0.189).</p>\r\n<p>\r\n	Table 2 showed the logistic regression analysis of the risk factors associated with peripheral diabetic neuropathy. Age and weight were the only factors found to be significantly associated with peri&shy;pheral neuropathy (age; p: 0.039; weight; p: 0.005).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	It is known that although a proportion of people with peripheral neuropathy present with pain and numbness, many are still asymptomatic until much later, hence it is imperative for physicians to identify those who are at risk to develop these complications. This study hig&shy;hlighted the fact that more than half of the DM cohorts demonstrated peripheral neuropathy, with age and body weight significantly associated with neuropathy.</p>\r\n<p>\r\n	Our results confirm the well-established correlation between age and peripheral neuropathy. This finding could be attri&shy;buted to two main factors. First, the du&shy;ration of DM is acknowledged to be as&shy;sociated with the development of compli&shy;cations of diabetes, and peripheral neu&shy;ropathy is no exception. Secondly, the development of peripheral neuropathy is also known to increase with age, espe&shy;cially after the age of 65 in the general population with or without diabetes (Cho et al. 2006; Mold et al. 2004). However the mean age for our DM cohorts was much lower than what was reported by Cho and Mold (Cho et al. 2006; Mold et al. 2004). Taking into consideration the high level of HbA1c amongst our DM co&shy;horts, we postulated that the combina&shy;tion of age, duration of diabetes and the poor glycaemic control might have con&shy;tributed to the accelerating occurrence of peri&shy;pheral neuropathy amongst our DM pa&shy;tients.</p>\r\n<p>\r\n	Our findings also demonstrated that heavier patients were more likely to suf&shy;fer from severe neuropathy compared to those who were less heavy. This finding is consistent with other studies which showed that obese patients achieved poorer results in test of clinical neuropa&shy;thy compared to those who were less obese (Cavanagh et al. 1991; Straub et al. 1994). Further analysis showed that the amount of visceral fat was propor&shy;tionally related to the increase of overall insulin resistance. This could explain the findings of severe neuropathy in sub&shy;jects with heavier body weights. How&shy;ever, this finding was based on the weight alone not the body mass index (BMI) to give an accurate relationship between neuropa&shy;thy and level of obesity amongst DM pa&shy;tients.</p>\r\n<p>\r\n	Callus or hyperkeratosis has been im&shy;plicated in the etiology of neuropathic ulceration. Callus acts like a foreign body which in turn increases the likelihood for tissue breakdown and consequently for&shy;mation of an ulcer under the callus. Stu&shy;dies have shown that plantar calluses were a strong predictor for ulcers and ulcers recurred mainly at the site of cal&shy;luses (Murray et al. 1996; Menz et al. 2007). In the present study, the presence of callus was found to be associated with higher NDS. This is important as simple measures like paring of the callus may reduce the risk of plantar ulceration (Young et al. 1992). The fact that cal&shy;luses were present in approximately 30% of the cohort, suggested that poor foot care was common among our patients and this warrants further intervention in both patients and health practitioners</p>\r\n<p>\r\n	We were not able to demonstrate any associations of neuropathy with the male gender, ethnicity and duration of diabetes mellitus. Peripheral neuropathy was also not significantly related to smoking, but most of our patients were non-smokers. However it is known that smoking causes the arrest of blood flow in the peripheries which in turn causes tissue hypoxia and neural microvascular damage leading to the development of peripheral neuropa&shy;thy (Benbow et al. 1997). The glycaemic control was not independently associated with neuropathy; the reason may be the cross sectional design of this study and a single and latest HbA1c result is possibly a poor indicator of the overall diabetic control.</p>\r\n<p>\r\n	This study provided information to practitioners in identifying potentially re&shy;versible factors contributing to the devel&shy;opment of peripheral neuropathy, namely the patient&rsquo;s body weight and presence of callus. It supported the deemed im&shy;portance of weight reduction in Type 2 diabetes patients in preventing progres&shy;sion of neuropathy and consequently the costly complications of plantar ulceration and lower extremity amputation. This study also underlined the importance of implementing a proper foot care program especially for those who are heavier and older. Although these prac&shy;tices have already been recommended by many clinical guidelines, many practi&shy;tioners fail to adhere to them and fail to identify those at risk of peripheral neuro&shy;pathy. So, an increased awareness by primary care providers of the associated risk factors can lead to an early thera&shy;peutic intervention and prevention of later neu&shy;ropathic complications such as infec&shy;tion and foot ulcers, allowing a decrease in patient morbidity.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	This study demonstrated that age and weight were the risk determinants for the development of peripheral neuropathy. The presence of calluses was also found to be associated with those with peri&shy;pheral neuropathy. Early detection of these risk factors should be incorporated into any program aiming to reduce di&shy;abetic foot complication as to reduce morbidity that is commonly associated with diabetes mellitus. However, the study is limited by its cross-sectional de&shy;sign, we were unable to assess the cause and effect of the study. Likewise, population drawn was from an outpatient setting in a university hospital which in turn causes selection bias in this study. Patients in our cohort might be substan&shy;tially different from a community-based outpatient setting as our out-patient clin&shy;ics are mainly referral-based; hence it might not be a true representative of the general population. Nonetheless, despite the limitations, this population-based sur&shy;vey was able to inform us regarding the diabetes foot problems in Malaysia, using inexpensive and simple assessments that can be replicated even in remote settings.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	We thank the patients and carer who contributed to the study. We also thank all the doctors and staff in the Medical Endocrine Clinic, UKMMC and UKM Pri&shy;mary Care Centre for their help during the conduct of the study.</p>\r\n', 1),
(446, 443, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Acute mastoid abscess usually occurs from the contagious spread of acute otitis media (AOM) to the mastoid portion of the temporal bone. Subclinical infection re&shy;sulting in mucosal oedema, will not mani&shy;fest clinically, is only seen as clouding of the mastoid air cells on CT scan. The in&shy;cidence reaches 100% in children with uncomplicated AOM (Lee 2008).Acute mastoid abscess most frequently occurs as result of partially or untreated cases of AOM in children. In the early 20th century 50% of all cases of otitis media developed a coalescent mastoiditis. By 1959, the incidence had fallen to 0.4% and currently the incidence is only 0.24% (Spiegel et al. 1998). According to Friedrich Bezold (1824-1908), 20% of patients with mas&shy;toiditis developed mastoid abscess (Spiegel et al. 1998).</p>\r\n<p>\r\n	In the presence of clinical mastoiditis, the infection from the mastoid air cells extends through the mastoid cortex re&shy;sulting in a mastoid abscess. Examination of the ear will show an obvious fluctuant swelling over the post auricular and ex&shy;tending to the suprauricular area. The skin over EAC is swollen and inflamed. In a mastoid abscess, infecting organisms are similar to those found in most cases of AOM, with <em>streptococcus pneumonia</em>and <em>haemophilus influenza</em>being most com&shy;monly isolated (Ludman &amp; Wright 1998). The occurrences of acute mastoiditis and acute mastoid abscess have reduced considerably with the use of better antibi&shy;otics. However, in the developing coun&shy;tries, these infections still remain major challenges with respect to diagnosis and management (Dubey &amp; Larawin 2007). In the presence of an abscess, surgery should be conducted, optimally within hours of diagnosis, which include post auricular incision and drainage, simple mastoidectomy and drainage of the wound (Nadol &amp; McKenna 2005). Here, we report a case of acute mastoid ab&shy;scess secondary to partially treated upper respiratory infection.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 7 year-old Malay girl, presented with a history of upper respiratory tract infection (URTI) of two weeks duration, associated with left otalgia and otorrhoea. She was partially treated with oral antibiotics and ear drops by two general practitioners (GPs) but the symptoms persist. Three days prior to admission, the otalgia wors&shy;ened and she developed left post auricular swelling. She denied any history of headache, photophobia, neck stiffness or nausea to suggest meningitis.</p>\r\n<p>\r\n	On examination, patient was febrile with a temperature of 39<sup>o</sup>C. However, she was not in distress. Purulent discharge was noted in the left external ear canal. There was a diffuse left posterior auricu&shy;lar swelling measuring about 2 x 3 cm. The swelling was fluctuant and tender and the overlying skin was inflamed. Ex&shy;amination under the microscope revealed sagging of the posterosuperior wall of the external ear canal. The tympanic mem&shy;brane was inflamed and thickened. There was pus oozing through the small central perforation. Hearing threshold was not performed as she had severe otalgia. She was treated with broad spectrum antibiotics and underwent an emergency left cortical mastoidectomy. Intraopera&shy;tively, there was an abscess in the mas&shy;toid cavity and the aditus was filled with granulation tissue. The tegmen mastoidii and posterior cranial wall adjacent to the sigmoid sinus were dehiscent. She was prescribed cefuroxime syrup on dis&shy;charge. During the last visit following a year after surgery, she was well and the wound healed well. She was asympto&shy;matic, the tympanic membrane was intact and pure tone audiometry showed nor&shy;mal hearing bilaterally.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	AOM implies a suppurative process of the middle ear space characterised by otalgia with evidence of middle ear effu&shy;sion, associated with symptoms of fever, anorexia, vomiting and diarrhoea. AOM is one of the commonest diseases of child&shy;hood. About 19% to 62% of children have had at least one episode of AOM by one year of age, and as many as 85% of them have had one episode of AOM by three years of age (Lee 2008). Peak inci&shy;dence is during the second half of the first year of life. The risk of the children to acquire AOM is considered high as per statistical data. The general population may not be aware about the disease and the children may present predominantly with complaint of fever. They are usually treated with antipyretics, analgesics and may or may not be given oral antibiotics. Non-compliance to medication is also rampant. The parents administer the full course of oral antibiotics once their chil&shy;dren were getting better. All these factors will contribute to untreated or under-treatment of the AOM which subse&shy;quently will lead to complications namely acute mastoiditis and mastoid abscess.</p>\r\n<p>\r\n	In this report, the patient presented with the clinical picture of AOM as a result of URTI. Though she had been given a few courses of oral antibiotics but this was not sufficient to arrest the infection into the mastoid air cells. Once the infection involved the mastoid air cells, suppura&shy;tion had taken place leading to a mastoid abscess. The standard treatment for mastoid abscess described in most Otol&shy;ogy text books is cortical mastoidectomy (Nadol &amp; McKenna 2005). A study had been performed by Al-Serhani (1998), comparing between cortical mastoidec&shy;tomy and incision and drainage (I &amp; D) with myringotomy in 10 patients with mastoid abscess following an AOM. Both groups (5 patients on each group) had received systemic antibiotics. They re&shy;ported the group that had been treated with I &amp; D had a smooth recovery without mastoidectomy (Al-Serhani 1998). How&shy;ever, they suggested that I &amp; D be done on inpatients with close observation to detect any deterioration, relapse or in&shy;tracranial complications. In this patient, cortical mastoidectomy was the treatment of choice as there was a mastoid ab&shy;scess.</p>\r\n<p>\r\n	Currently, a few groups had reported in their studies that chronic suppurative oti&shy;tis media (CSOM) can be the source of infection for development of acute mas&shy;toiditis and mastoid abscess. But most of these patients had an associated cho&shy;lesteatoma (Al-Serhani 1998).</p>\r\n<p>\r\n	Inadequate diagnosis and treatment are factors that contribute to the high preva&shy;lence of AOM in children in our practice. Cortical mastoidectomy is the treatment of choice along with systemic antibiotics. In centres without an Otorhinolaryngolo&shy;gist, I &amp; D with systemic antibiotics and close observation for early detection of complications may be carried out. The primary aim of the treatment in acute oti&shy;tis media is to prevent complications such as mastoid or intracranial abscess.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	General practitioners played an important role in managing a child with AOM in order to prevent complications. In this case, there was incomplete course of antibiotic given to the patient. Therefore we advocate that all AOM patients should be treated with antibiotics for a longer duration and to have early ENT referral.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(447, 444, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	The t(8;21)(q22;q22) is a frequently oc&shy;curring aberration in acute myeloid leu&shy;kemia (AML) (18-20%), and usually cor&shy;relates with the French-America-British (FAB) M2 subtype (Shinagawa et al. 1999). The translocation involves the <em>RUNX1 </em>gene on chromosome 21q22 and the <em>RUNX1T1</em> gene on chromosome 8q22. The translocation resulted in fusion of transcripts <em>RUNX1/RUNX1T1</em> on the de&shy;rivative of chromosome 8. It is re&shy;ported that patients with AML carrying the t(8;21)(q22;q22) exhibit good re&shy;sponse to chemotherapy with a high re&shy;mission rate and long-term disease-free survival. However, it may be associated with a high incidence of early relapse in some cases (Shinagawa et al. 1999). Trisomy 4 is a recurrent but rare chromo&shy;some abnormality which ap&shy;pears to oc&shy;cur in a broad range of he&shy;matologic ma&shy;lignancies, mainly cases of AML with FAB M2 and M4 subtypes, and some cases with M1 and M5 subtypes (Kwong et al. 1993; Shinagawa et al. 1999; Beghini et al. 2000). Trisomy 4 could also be second&shy;ary to an environmental mutagen or drug exposure (Shinagawa et al. 1999; Nishii et al. 2003). It may also present with a sole cytogenetic abnor&shy;mality or con&shy;comitantly with other aber&shy;rations. The significance of its occur&shy;rence is still unde&shy;fined.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	We report a case of a 33-year-old fe&shy;male presenting with fever and anemia. Her full blood picture (FBP) showed pancyto&shy;penia. The mor&shy;phology of the circulating blasts showed presence of Auer rods with hypergranu&shy;lation. Full blood count showed a white blood cell (WBC) count of 2.3 &times; 10<sup>9</sup> /L, with a hemoglobin (Hb) concentration of 6.4 g/L and a platelet count of&nbsp; 37&times; 10<sup>9</sup> /L. There was no his&shy;tory of long term medication and toxic chemical exposure. She was suspected as having AML, FAB M3 (APL). How&shy;ever, the bone marrow aspirate (BMA) was reported as only AML. BMA was sent for cytogenetic analysis to confirm the diag&shy;nosis. A trans-retinoic acid (ATRA) re&shy;gimen was given to her as the initial treatment. She died soon after the diag&shy;nosis.</p>\r\n<p>\r\n	<strong>CYTOGENETICS ANALYSIS</strong></p>\r\n<p>\r\n	Chromosome analysis of bone marrow aspirate was performed using the stan&shy;dard cytogenetic protocol. The karyotype was described according to the Interna&shy;tional System for Human Cytogenetic Nomenclature (Shaffer &amp; Tommerup 2005). Thirteen metaphases were ana&shy;lysed. Cytogenetic analysis demon&shy;strated 46,XX,+4, t(8;21)(q22;q22) (Fig&shy;ure 1). This finding excluded the di&shy;agno&shy;sis of AML M3, but indicated that the pa&shy;tient is having AML with trisomy 4 in ad&shy;dition to t(8;21).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The t(8;21)(q22;q22) AML present at di&shy;agnosis is associated with a better prog&shy;nosis and generally demonstrated pro&shy;longed disease-free survival (Nishii et al. 2003). AML with the t(8;21) is frequently associated with a loss of sex chromo&shy;some Y in males and an inactive X in fe&shy;males, and 3.4% of the cases are variant translocations (Shinagawa et al. 1999; Nishii et al. 2003). A case of FAB M2 subtype with anomalies, complex trans&shy;location of t(6;21;8) and trisomy 4 have been reported. However, the presence of trisomy 4 has not demonstrated any in&shy;fluence in clinical prognosis (Shinagawa et al. 1999).</p>\r\n<p>\r\n	Aberrations in chromosome 7 and chromosome 8 are reported to be asso&shy;ciated with secondary AML or during clonal progression of AML (Beghini et al. 2000; Trivedi et al. 2008). Trisomy 4 is rarely reported as the ab&shy;normality in AML (&lt;1%) (Trivedi et al. 2008). Nishii et al<em>.</em> (2003) showed that t(8;21) AML with trisomy 4 is very rare. Out of 94 cases, only three were found to have concomi&shy;tant aberrations of t(8;21) with trisomy 4 (3.2%) (Nishii et al. 2003). t(8;21) AML with trisomy 4 has a poor prog&shy;nosis and was speculated that the presence of <em>KIT </em>mutation may be responsible for the ad&shy;verse effect of the disease (Langabeer et al. 2003; Trivedi et al. 2008). Few stu&shy;dies have demonstrated that patients with the t(8;21) and a trisomy 4 showed a signifi&shy;cantly high frequency of<em>KIT </em>muta&shy;tion (10%) than patients with normal and complex aberrant karyotype (Shimada et al. 2006). Patients with activation of <em>KIT </em>gene have been shown to confer drug resistance, which could explain the rela&shy;tively poor prognosis (Beghini et al. 2000; Langabeer et al. 2003; Shimada et al. 2006). <em>KIT</em> is localized at chromosome 4q12, thus trisomy 4 leads to an in&shy;creased gene dosage of <em>KIT</em>. It is be&shy;lieved that the occurrence of t(8;21) represents the initial step to initiate the disease, followed by activation of <em>KIT</em> pathway that promotes disease progres&shy;sion (Beghini et al. 2000; Shimada et al. 2006).</p>\r\n<p>\r\n	In our case, the patient apparently had clinical and hematological features sug&shy;gestive of acute promyelocytic leukemia (APML) in whom the cytogenetic analysis revealed the presence of trisomy 4 with an additional t(8;21) hence, ex&shy;cluding the di&shy;agnosis of APML. Inclu&shy;sion of cytogenetic analysis in addition to clinical and morpho&shy;logical criteria will pro&shy;duce a reliable clinical diagnosis of the dis&shy;ease. This is because of variable morphol&shy;ogy due to the presence of complex or ad&shy;ditional chromosomal al&shy;teration, also the multiple genetic events which may affect the vari&shy;ous maturation stages of cells. Thus, cytogenetic analysis is one of the useful tools to confirm the clinical diagno&shy;sis. Detection of the t(8;21) can be further investigated with florescence in situ hy&shy;bridization (FISH)&nbsp; analysis to de&shy;termine the <em>AML1</em> gene rearrangement. Other di&shy;agnostic tests include immunophe&shy;no&shy;typing which may help to identify the subtype of t(8;21) AML.</p>\r\n<p>\r\n	A study by Nishii et al. (2003) reported that most of the cells carrying trisomy 4 showed low expressions of CD19 and IL-17 receptor, and high expressions of CD33, CD18, and CD 56 when com&shy;pared to t(8;21) AML without other addi&shy;tional chromosomal abnormality (Nishii et al. 2003). CD56 expression in AML is reported in multidrug resistance (Trivedi et al. 2008). There was no flow cytomet&shy;ric analysis done in our case. There&shy;fore, we cannot explain the relation be&shy;tween CD56 expression and trisomy 4 in t(8;21) AML.</p>\r\n<p>\r\n	&nbsp;In conclusion, the present case of AML FAB M2 is a rare case in terms of cyto&shy;genetic findings. Concomitant t(8;21) with trisomy 4 may constitute a distinctive subtype of t(8;21) AML (Nishii et al. 2003). Hence the acquisition of trisomy 4 in AML with <em>AML1</em> rearrangement might promote disease progression and an unfa&shy;vourable prognosis. This case illu&shy;strated the essential roles of cytogenetics studies in confirming the diagnosis. Cy&shy;togenetics analysis provides simultane&shy;ous analysis of genetic rearrangements within leukemic cell. Meanwhile FISH analysis approached is a complementary test for conventional cytogenetics that helps to determine the chromosomal rearrangements such as t (8;21), t(15;17) and inv(16) in AML cases.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	We would like to thank the Director Gen&shy;eral of Health Malaysia for permission to publish this paper. We would like to ac&shy;knowledge the Director of Ampang Hos&shy;pital, Datin Dr Aishah bt Ahmad Mukinuddin, members of IMR&rsquo;s genetic laboratory for their technical as&shy;sistance and staff of the Haematology De&shy;partment, Hospital Am&shy;pang for their sup&shy;port.</p>\r\n', 1),
(448, 445, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Primary thymic lesions such as thymic cyts, hyperplasia, carcinoma, and thy&shy;momas comprise approximately 2-3% of all pediatric mediastinal tumours (Grosfeld 1994). Thymoma is a tumour originating within the epithelial cells of the thymus. Patients can present with chest pain, cough, dyspnoea, dysphagia, hoarseness, superior vena cava syn&shy;drome or paraneoplastic syndrome in&shy;cluding myasthenia gravis, pure red cell aplasia, or acquired hypogammaglobuli&shy;naemia, and connective tissue disease disorders. Most patients are asympto&shy;matic. Thymoma can generally be cate&shy;gorised as non-invasive (Stage I) and invasive (Stage II-IV) (Masaoka et al. 1981). Treatment of thymoma includes surgery, chemotherapy, and irradiation. Surgical resection is the treatment of choice for stage I and stage II tumours. In more advanced diseases, systemic therapy has shown to give a response rate of 50% to 80% (Thomas et al. 1999). Therefore, a multimodal therapy is often use for the treatment of patients with ad&shy;vanced thymoma. Stage of the tumour is an independent prognostic factor for sur&shy;vival.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 16-year-old boy was evaluated for cough and chest pain. His past medical history was unremarkable. On physical examination, the patient showed signs of mild superior vena cava (SVC) obstruc&shy;tion, with decreased right chest breath sounds. Other physical examination findings were normal. An initial chest X-ray showed a huge mass in the anterior mediastinum. Computerized tomography (CT) of the thorax showed an anterior me&shy;diastinal mass with a diameter of 6x10x11cm that abutted the great ves&shy;sels and had posteriorly displaced and com&shy;pressed the SVC (Figure 1). The pa&shy;tient underwent a CT-guided percutane&shy;ous biopsy of the mediastinum mass as it was considered inoperable. Histopatho&shy;logical examination revealed a neoplasm composed of epithelial cells with sur&shy;rounding lymphoplasmacytic infiltra&shy;tion (Figure 2). The neoplastic epi&shy;thelial cells display round to oval vesicu&shy;lar neuclei. Immunohistochemically, the neoplastic cells were positive for cytoke&shy;ratin and negative for LCA, vimentin, and S-100, confirming the diagnosis of thy&shy;moma.</p>\r\n<p>\r\n	Neoadjuvant chemotherapy was started to improve resectability of the tumour mass. A chemotherapy protocol contain&shy;ing cisplatin (50mg/m2/day, for 1 day), cyclophosphamide (500mg/m2/day, for 1 day), and doxorubicin (50mg/m2/day, for 2 days) was given and repeated every 21 days. After 3 cycles of chemotherapy,&nbsp; about 50% reduction in tumour size was observed on a repeat CT scan of the tho&shy;rax (mass size: 5.4x4.6x7.5cm). Subse&shy;quently, he was scheduled for a tumour excision, however, the surgery was per&shy;formed after the 4th cycle of chemo&shy;the&shy;rapy. At the time of median sternot&shy;omy, the mass was densely adhered to the underlying pericardium, brachio-ce&shy;phalic vein, SVC, and right upper lobe of the lung. The tumour had completely en&shy;cased the right phrenic nerve and had infiltrated the wall of the SVC anterolate&shy;rally and the wall of the brachio-cephalic vein anteriorly. Incomplete resection of the tumour was performed together with resection of the involved pericardium. A small rim of the tumour tissue that had infiltrated the SVC and the brachio-ce&shy;phalic vein walls could not be excised. Final pathology showed thymoma with unclear margins, consistent with the World Health Organization (WHO) classi&shy;fication of thymoma type B2 and the Ma&shy;saoka classification of stage III.&nbsp;</p>\r\n<p>\r\n	Post-operatively the patient received two cycles of chemotherapy; however, a resi&shy;dual tumour of 2.0x1.0x0.5cm was seen on a repeat CT scan of thorax. Subse&shy;quently, the patient was treated with 30Gy radiotherapy in 15 fractions at 2.0 Gy per fraction to the tumour area followed by boosts of 16Gy in 8 fractions at 2.0 Gy per fraction over a period of 4 weeks. The tumour had cleared on re&shy;peat CT scan of thorax at the end of the treatment. The patient has been in re&shy;mission for 6 years without radiographic evidence of tumour recurrence.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Thymoma is uncommon in children. Therefore, it is important to recognise this tumour as distinct from others like mediastinal teratoma, lymphoma, malig&shy;nant histiocytoma and Ewing sar&shy;coma/primitive neuroectodermal tumour. Most thymomas are confined to the me&shy;diastinum at the time of diagnosis. Dis&shy;tant metastasis is rare.</p>\r\n<p>\r\n	The widely accepted staging system in use for thymoma is that proposed by Ma&shy;saoka et al. (1981), based on a post-operative staging procedure, since cap&shy;sular invasion, a key component of this staging system, is best evaluated by pa&shy;thologic examination. Recently, the WHO classification of thymic tumours (Rosai &amp; Sobin 1999) based on the histological assessment of the morphology of the neoplastic epithelial cells has received increasing acceptance, and it has been shown to be of prognostic significance (Chen et al. 2002; Kondo et al. 2004).&nbsp; Thymomas are classified as type A, AB, and B in the WHO classification, and ex&shy;hibit organotypic (thymus-like) architec&shy;tural features. Nevertheless, Kim et al. (2010) reported that the prognostic re&shy;levance among type B thymoma sub&shy;types is still controversial. They did not find any significant differences in the in&shy;cidence of recurrence and survival among the three subtypes of type B thy&shy;mo&shy;mas. Tumour recurrence was signifi&shy;cantly associated with advanced Ma&shy;saoka stages. Some have shown that patients with type B3 thymomas have worse prognoses than those with type B2 thymomas (Okumura et al. 2002; Nakagawa et al. 2003), whereas others have reported a similar survival rate be&shy;tween these two groups (Chen et al. 2002; Str&ouml;bel et al. 2004). Therefore, Masaoka stage should always be consi&shy;dered when predicting prognosis and planning treat&shy;ment, including adjuvant treatment for patients with type B thy&shy;momas.</p>\r\n<p>\r\n	Surgical resection is the mainstay of treatment in patients with resectable dis&shy;ease (Yagi et al. 1996; Wang et al. 1992). The extent of surgical resection remains the most important prognostic factor for predicting tumour relapse (Singhal et al. 2003). Dhall et al. (2004) reported two children with completely en&shy;capsulated thymoma who were suc&shy;cessfully treated with surgery alone, and remain free of disease 3 years after sur&shy;gery. However, multimodal therapy con&shy;sisting of chemotherapy, radiotherapy, and/or surgery is recommended for any incompletely resected &ldquo;high-risk&rdquo; thy&shy;moma, locally advanced or metastatic thymoma (Chahinian et al. 1981; Str&ouml;bel et al. 2005). It has been shown that mul&shy;timodal treatment of patients with neoadjuvant chemotherapy, and surgery, followed by additional adjuvant chemo&shy;therapy plus radiotherapy, may improve the survival of patients with locally ad&shy;vanced thymoma (Venuta et al. 1997; Hassan &amp; Seoud 2009). In addition, in&shy;duction chemotherapy may be effective in downstaging thymoma, allowing pa&shy;tients initially thought not to be surgical candidates to undergo resection.</p>\r\n<p>\r\n	Our patient had advanced tumour stage III Masaoka classification. The benefit of multimodal therapy was demonstrated in our patient as the patient showed a par&shy;tial response to the induction chemothe&shy;rapy improving the resectability of the mass during surgery. Although &quot;debulk&shy;ing&quot; or subtotal resection have been re&shy;ported for invasive thymoma, most mod&shy;ern day series have demonstrated long term survival correlating best with com&shy;plete surgical removal. Therefore, we believe most surgeons would likely per&shy;form en bloc great vein and phrenic nerve resection in this case.&nbsp; Removal of one brachiocephalic vein is usually well tolerated and there are a number of techniques to reconstruct the SVC. Di&shy;aphragmatic plication could also be per&shy;formed to improve breathing mechanics after phrenic nerve removal, which may be dysfunctional from tumour involve&shy;ment to begin with. Subsequently, the postoperative adjuvant chemotherapy and radiotherapy further help to clear the tumour in this patient. Previously, Shin et al. (1998) reported that induction che&shy;motherapy consisted of cyclophospha&shy;mide, doxorubicin, and cisplatin was given to patients with unresectable stage III and IV thymoma. Out of 11 patients initially thought to be unresectable, nine were able to undergo resection. All nine patients were given additional postoper&shy;ative adjuvant chemotherapy and radio&shy;therapy. Out of these patients, seven were disease-free at a median follow-up period of 43 months. Yokoi et al. (1992) reported a successful treatment with preoperative/postoperative cisplatin and doxorubicin, in addition to postoperative radiotherapy in a patient with a locally advanced thymoma invading the heart and great vessels. Macchiarini et al. (1991) reported that four patients with clini&shy;cal stage III thymoma received three cycles of cisplatin, epirubicin, and etopo&shy;side before surgery, and later achieved a complete remission. Rea et al. (1993) reported that 16 patients with stage III and IVa disease were treated with a dox&shy;orubicin, cisplatin, vincristine, and cyclo&shy;phosphamide regimen every three weeks for three to four cycles. After chemothe&shy;rapy, sur&shy;gery was performed, and if resi&shy;dual dis&shy;ease was present, postoperative radia&shy;tion therapy was given. Patients with a complete remission received three additional cycles of chemotherapy. The authors demonstrated seven complete and five partial responses and a pro&shy;jected two-year sur&shy;vival rate of 80%. Loehrer et al. (1997) reported a response rate of around 70% in 23 patients with stage III thymoma who received chemo&shy;radiotherapy. The pro&shy;gression-free and overall survival rates at five years were 54% and 52% respectively.&nbsp;</p>\r\n<p>\r\n	As demonstrated in this patient, the use of multimodal therapy in locally advanced thymoma was effective, significantly de&shy;creasing the tumour size, thus improving the patient&rsquo;s surgical outcome, and over&shy;all contributed to a good long-term out&shy;come.</p>\r\n', 1),
(449, 446, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Fungiform papilloma, inverted papilloma and oncocytic Schneiderian papilloma are three morphologically separate le&shy;sions arising from the Schneiderian membrane (Hyams 1971). Oncocytic type is the rarest which comprises about 3~5% and occurs mostly in patients over 50 years of age (Cunningham et al. 1987). Characteristic histological ap&shy;pearance of multilayered eosinophilic epithelium with mixed exophytic and in&shy;verted growth pattern differentiates this tumour from the other type of papillomas. The clinical be&shy;havior is similar to inverted papilloma but has a higher rate of local recurrence and malignant transformation (Kaufman et al. 2002).</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 78 year old lady presented with recur&shy;rent unilateral epistaxis associated with progressive nasal blockage for six years. She denied facial numbness or visual symptoms. Clinically there was no op&shy;thalmoplegia. Nasal endoscopy showed a friable mass arising from the left middle meatus filling the nasal cavity and naso&shy;pharynx (Figure 1). Otherwise, ear, throat, head and neck examination was unre&shy;markable.</p>\r\n<p>\r\n	An intranasal biopsy was taken which showed histological features of oncocytic papilloma. Computed tomography of the paranasal sinuses showed a soft tissue mass filling the left maxillary antrum, ex&shy;tending into the nasal cavity and naso&shy;pharyx with widening of the maxillary ostium. There was no bony erosion noted which would suggest malignancy.</p>\r\n<p>\r\n	The patient underwent an endoscopic medial maxillectomy for tumour excision. Intraoperative findings revealed that the tumour mass was occupying the left na&shy;sal cavity with its attachment within the maxillary sinus. The surgery started with debulking of the tumour with a powered mi&shy;crodebrider until visualization of the tu&shy;mour protruding through the maxillary ostium. The procedure then entailed a radical inferior turbinectomy following which an L-shaped osteotomy was per&shy;formed. The vertical limb corresponded to the anterior wall of the maxilla and the horizontal limb corresponded to the floor of the maxillary sinus. The tumour was then removed using a microdebrider and its origin traced to the posteromedial wall of the maxillary sinus. Complete tumour excision was achieved with preservation of healthy surrounding mucosa.</p>\r\n<p>\r\n	Histopathological examination of the specimen revealed multiple polypoidal tissue with a central core of connective tissue composed of proliferating colum&shy;nar epithelial cells with admixture of mu&shy;cin containing cells and numerous micro&shy;cysts (Figure 2) which was consistent with the diagnosis of oncocytic papilloma. There was no evidence of malignancy in the multiple samples of the surgical spe&shy;cimen.</p>\r\n<p>\r\n	The patient had an uneventful post&shy;op&shy;erative recovery. She was last seen at six months post operatively and there was no recurrence noted.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Oncocytic papilloma (OCP) is derived from the Schneiderian membrane and is also called cylindrical cell papilloma or columnar cell papilloma (Hyams 1971). The etiology for OCP is unknown. There is no sex predilection, unlike in inverted and fungiform types which occur predo&shy;minantly in males.</p>\r\n<p>\r\n	The most common presenting features are unilateral nose block and intermittent epistaxis. OCP always occurs unilate&shy;r&shy;ally, usually arising from or involving the maxillary sinus (Barnes &amp; Bedetti 1984; Hyams 1971) in contrast to inverted pa&shy;pilloma which often arises from the lat&shy;eral nasal wall (Lawson et al. 1995).</p>\r\n<p>\r\n	Grossly, OCP appears as a fleshy pink papilloma or grey polypoidal growth. The classic histologic description of OCP is the presence of both exophytic and in&shy;verted growth patterns, composed of multilayers of columnar cells with eosi&shy;nophilic cytoplasm and small uniform dark nuclei. The epithelial cells of OCP are true oncocytes from the sinonasal respiratory epithelium - hence the term oncocytic Schneiderian papilloma was used. The epithelium characteristically contains small cysts filled with mucin or neutrophils (microabscess) (Barnes &amp; Bedetti 1984; Hyams 1971).</p>\r\n<p>\r\n	OCP has similar clinical behaviour with the inverted type which includes a pro&shy;pen&shy;sity for local invasiveness, recurrence and malignant transformation. Although OCP is relatively uncommon, they have a higher rate of recurrence and malignant transformation. The recurrence rate is 33% versus 15% and malignant change is 17% versus 9% in OCP compared to inverted papilloma (Kaufman et al. 2002). The most frequent type of malignancy is invasive squamus cell carcinoma. As in inverted papilloma, the carcinoma may arise within the papilloma as evidenced by a degradation of histologic changes ranging from dysplasia to carcinoma in situ to invasive carcinoma or simply a coexistence of carcinoma with papilloma at the same anatomical site.</p>\r\n<p>\r\n	Complete resection of this tumour is advocated in view of the high recurrence rate and possibility of missing an associ&shy;ated carcinoma. Much has been written on the advantages of endoscopic medial maxil&shy;lectomy over the external approach. En&shy;doscopic approach has superior mag&shy;nifi&shy;cation and angled view for better visu&shy;ali&shy;zation and delineation of tumour mar&shy;gins. Endoscopic sinus surgery including en&shy;doscopic medial maxillectomy is a vi&shy;able primary treatment alternative for si&shy;no&shy;nasal inverted papilloma with compa&shy;rable recurrence rates to external ap&shy;proaches (Wormald et al. 2003; Nicolai et al. 2006). Tumours arising from an&shy;teromedial and anterior wall of the max&shy;illary sinus can be addressed by an en&shy;doscopic medial maxillectomy in which removal of the medial wall of the maxilla (including the inferior turbinate) up to its anterior wall is performed. It can be com&shy;bined with a canine fossa puncture to resect disease arising from the lateral wall and floor of the maxillary sinus (Wormald et al. 2003). It can also provide excellent and com&shy;plete exposure of the maxillary sinus and ethmoids without re&shy;moving the lamina papyracea, the medial floor of the orbit, the anterior maxillary wall, and the frontal process of maxilla with preserva&shy;tion of the medial palpebral ligament, unlike open medial maxillec&shy;tomy (Sadeghi et al. 2003). Lack of an external scar, shorter operative time and reduced bleeding re&shy;duces morbidity and hospital stay. How&shy;ever, an open ap&shy;proach is advocated in tumours extend&shy;ing into the orbit, frontal sinus and intra&shy;cranium as well as those with wide at&shy;tachment within the sinonasal complex (Tufano &amp; Thaler 1999).</p>\r\n<p>\r\n	In our patient the endoscopic approach was opted in view of the limited tumour extension which was viable for complete excision via the endoscopic technique. Preoperative imaging was also not sug&shy;gestive of any associated malignancy where an open approach would be advo&shy;cated. Endoscopic tumour excision also affords shorter hospital stay with no as&shy;sociated morbidity which was ideal in our elderly patient.</p>\r\n<p>\r\n	Post operative routine endoscopic sur&shy;veillance is important regardless of the surgical approach to detect early recur&shy;rence of tumours that may be harbouring a malignancy.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Oncocytic papilloma is uncommon and has a higher rate of recurrence and ma&shy;lignant transformation compared to in&shy;verted papilloma. It should be considered in the differential diagnosis of unilateral polypoid nasal lesions especially in el&shy;derly patients. Endoscopic medial maxil&shy;lectomy can be the procedure of choice as it is minimally invasive and provides good tumour clearance in selected cases.</p>\r\n', 1),
(450, 447, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	<em>Ascaris lumbricoides </em>is one of the soil-transmitted helminthes (STH) and is known to be the largest intestinal round&shy;worm of humans. It is a cosmopolitan intestinal parasite with a high prevalence in poor and socio-economically deprived communities in the tropical and sub-tropi&shy;cal areas and is more common in child&shy;ren. About 25% of the world&#39;s popula&shy;tions are infected by <em>A. lumbricoides</em> and this causes up to a million cases of the disease annually. Poverty, overcrowded living conditions, poor environmental sa&shy;nitation and lack of health education are the factors related to the intestinal hel&shy;minthic infection (Norhayati et al. 2003). Several studies have demonstrated a high prevalence of <em>A. lumbricoides </em>infec&shy;tion among Orang Asli (Aborigines) child&shy;ren in Malaysia (Norhayati et al. 1997; Mohamed Kamel et al. 2001; Hesham Al-Mekhlafi et al. 2007) where the preva&shy;lence of ascariasis ranged between 33.3% - 69%.</p>\r\n<p>\r\n	The objectives of this study were to determine the prevalence of <em>A. lumbricoides </em>infection among 1-12 years old Orang Asli children in Pos Lenjang, Pahang and to evaluate the outcome of the concentration technique compared to direct faecal smear in detecting <em>Ascaris</em> infection.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	<em>Subjects and study area </em></p>\r\n<p>\r\n	Pos Lenjang is an aboriginal settlement in Pahang, situated about 320 km from Kuala Lumpur. The aborigines are from the Semai tribe and the village comprises a very small population. After an in&shy;formed consent was obtained, stool containers were distributed to the popu&shy;lation. A total of 71 faecal samples were collected from children aged between 1-12 years old of which 31 were boys and 40 girls.</p>\r\n<p>\r\n	<em>Faecal parasitological examination</em></p>\r\n<p>\r\n	Each faecal sample was subdivided into two parts: one part was unfixed (fresh sample) and the second part was fixed with 10% formalin.&nbsp; Both types of sam&shy;ples were examined for the presence of <em>A. lumbricoides</em> ova. The fresh samples were screened immediately in the field using the direct faecal smear technique and two smears were made from each sample. Meanwhile, the formalin-fixed samples were examined later using the formalin-ether concentration technique at the Parasitology Laboratory, Department of Biomedical Science, Universiti Ke&shy;bangsaan Malaysia</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Seventy one faecal samples were ex&shy;amined using direct faecal smear and formalin-ether concentration techniques. Out of 71 children, 30 (42.3%) were found to be positive for <em>A. lumbricoides</em>. The prevalence of <em>Ascaris</em> infection was higher (45.2%) in males compared to females (40%) (p&gt;0.05). A higher preva&shy;lence was also found among the school-aged children (48.6%) as compared to the preschool children (35.3%) (p&lt;0.05) (Table 1).</p>\r\n<p>\r\n	Of the 30 positive samples, the formalin-ether concentration recorded a detection rate of 100%&nbsp; (30 of 30) while the direct faecal smear technique detected only&nbsp; 46.7% (14 of 30) of the total positive samples (p&lt;0.05)(Table 2).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The overall prevalence of ascariasis in this study was 42.3% which revealed that <em>A. lumbricoides</em> infection was common among the Orang Asli children in Pos Lenjang, Pahang. This finding is in agreement with previous studies done in Malaysia where the overall prevalence of <em>Ascaris</em> in 1-12 year-old Orang Asli child&shy;ren reported were 62.9% (Norhayati et al. 1997) and 59.5% (Rahmah et al. 1997). It should be noted that these studies were done at different study areas. Two more recent studies conducted among 2-15 year-old Orang Asli children from different study areas have reported similar findings with 61.9% prevalence of a<em>scariasis </em>(Hesham Al-Mekhlafi et al. 2006; Nor Aini et al. 2007).&shy; In other local studies involving the Orang Asli commu&shy;nity where all age groups were included, the results showed a 33.3% (Mohamed Kamel et al. 2001) and 25.7% (Lokman Hakim et al. 2007) prevalence rate of the infection.</p>\r\n<p>\r\n	According to Zulkifli et al. (2000), the prevalence of the STH infection is closely related and dependent on socio-eco&shy;nomic and hygiene conditions.During the study, the Orang Asli&rsquo;s ac&shy;tivities, basic facilities and environmental conditions were observed.Like other disadvantaged communities in Malaysia, the Orang Asli at Pos Lenjang also belongs to a lower socioeconomic group.&nbsp; Be&shy;cause of the poor educational level of the villagers, they work as farmers, animal hunters and traders of forest products. Due to low family income, the children live in poor-conditioned houses. Most of the houses in this village were inhabited by large families. This overcrowded living condition may enhance the faecal&ndash;oral transmission among the children in this community.</p>\r\n<p>\r\n	Poor personal hygiene was observed among the Orang Asli children due to the lack of knowledge about health and hy&shy;giene practices. Hand washing before eating and after defecation was not reg&shy;ularly practiced by the children. They also ate raw food especially fruits without washing them first. The disease trans&shy;mission may be enhanced among this population group who fail to practice good personal hygiene. Based on the review prepared by Fung &amp; Cairncross (2009), there was some evidence impli&shy;cating that the practice of hand washing is effective in reducing the prevalence of <em>Ascaris </em>infection.</p>\r\n<p>\r\n	Lack of basic facilities such as safe drinking water, proper housing and proper sanitary and garbage disposal render the Pos Lenjang community es&shy;pecially the children vulnerable to <em>Ascaris</em> infection. There was very limited number of stand pipes provided in this Orang Asli village. Several families had to share one pipe for their needs and therefore, the existing stand pipes could not supply enough water to all families in the village. Therefore they had to resort to alterna&shy;tive sources and the river played an im&shy;portant role as a source of water for them. They used water from the river for daily activities such as washing clothes, bathing, cooking and drinking. Toilets were not available and generally the vil&shy;lagers, especially children, were inclined to defecate indiscriminately among the bushes close to their homes. Besides bushes, some of the villagers used the river as a defecation site and using river water to clean themselves. The improper disposal of excreta practiced by this community will increase the risk of <em>Ascaris</em> infection among the children. A number of studies have reported that in&shy;adequacy of safe water supply (Norhayati et al. 1998) and poor sanitary disposal (Norhayati et al. 1998; Hesham Al-Mekhlafi et al. 2007) were the signifi&shy;cant risk factors for ascariasis in Orang Asli communities. According to Luong (2002), the optimum development of <em>Ascaris</em> occurs in moist warm soils where the eggs develop into an infective stage. The conditions in the Pos Lenjang area may be favourable for the development of <em>Ascaris</em>. The soil is suitable for hel&shy;minth&acute;s egg survival and transmission since there are many shady areas from the presence of many trees contributing to the high prevalence of <em>Ascaris</em> infec&shy;tion among the children in this commu&shy;nity.</p>\r\n<p>\r\n	This post has no accessible road for the provision of adequate garbage disposal. Hence, piles of garbage were left ex&shy;posed and unattended which attracted flies, cockroaches and other pests. A re&shy;cent study showed that the housefly is a potential mechanical vector for STH in&shy;fection, which could spread the infection by transmitting the infective eggs of hel&shy;minthes to the community with poor sa&shy;nitary conditions (Maipanich et al. 2008).</p>\r\n<p>\r\n	Based on the review carried out by Kan (1988), there was little difference in the distribution of helminthes infection be&shy;tween genders, especially boys and girls as they share similar socio-behavioral activity. In this study, males showed a slightly higher prevalence of infection at 45.2% as compared to females 40%, which were not significant statistically.&nbsp;</p>\r\n<p>\r\n	The result of this study showed that the highest prevalence of <em>Ascaris</em> infection was among the schoolchildren (48.6%) as compared to preschool children (35.3%). Bundy et al. (1988) and Norhayati et al. (1997) have reported that the prevalence of <em>Ascaris</em> was signifi&shy;cantly increased with age. Results from another study performed on children in Gombak by Rajeswari et al. (1994) showed that the prevalence of <em>Ascaris</em> increased reaching a peak in the 9-year age group (15.2%). Sagin et al. (2002) also reported that <em>Ascaris</em> infection was more common among children aged 6-14 years than in children below 6 years old who lived in the Bakun Valley, Sarawak. The possible reasons for the obvious difference in the preva&shy;lence between age groups are probably due to the children&acute;s habits and exposure. Children aged 7-12 years spend more time out of the house. Therefore, they have higher chances of exposure to the infective agent especially when playing on contaminated soil. On the other hand, the younger preschool children are usually under constant su&shy;pervision by their parents and spend more time indoors thus reducing expo&shy;sure.&nbsp;</p>\r\n<p>\r\n	In this study, the faecal specimens were examined for the presence of <em>Ascaris</em> ova by two techniques namely the direct smear and the formalin-ether concentration.&nbsp; Based on the results, the formalin-ether concentration was found to be the more sensitive in detecting <em>Ascaris</em> ova in faeces with a detection rate of 100% as compared to 46.7% with the di&shy;rect smear (p&lt;0.05). In this study, two faecal smears were made from the single stool specimen collected, the result could be different if more stool samples were examined for direct faecal smear. A pre&shy;vious study (Oguoma &amp; Ekwunife 2007) reported that 9.47% of <em>A. lumbricoides</em>was detected by the concentration technique while 5.26% was detected by the direct smear. They suggested that the concen&shy;tration technique is a very useful tech&shy;nique in diagnosing intestinal helminthes since it depicted a higher percentage of helminthes infection missed by the direct smear technique. Thus, it is recom&shy;mended that both techniques (the direct smear and concentration techniques) be performed in routine parasito&shy;logical examination for a more accurate diagnosis before any control efforts be implemented in the affected areas.</p>\r\n<p>\r\n	<em>Ascariasis </em>is still very prevalent among the Orang Asli children at Pos Lenjang, Pahang and it is one of the major causes of public health problems among Orang Asli communities in Malaysia that needs to be addressed.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	We would like to thank Ms. Shafariatul Akmar, Mr. Rahman from Parasitology Laboratory, Universiti Kebangsaan Ma&shy;laysia for their field and technical assis&shy;tance. We would also like to express our gratitute to the Kuala Lipis Health and Orang Asli Department for their invalua&shy;ble assistance and support throughout this study.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(451, 448, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Cervical cancer is the second most common cancer among women worldwide. The WHO (2002) reported that in 2002, over 471,000 new cases were diagnosed and 288,000 women died from cervical cancer worldwide. The incidence and mortality rate of cervical cancer were higher in developing countries (Sankaranarayanan et al. 2001), where no effective, systematic and organised screenings are carried out. It is recommended that for a screening method to be effective, it must be targeted on high risk women once or twice in their lifetime using a sensitive method (Sankaranarayanan et al. 2001) and a high enough coverage of more than 80% of targeted population.</p>\r\n<p>\r\n	The Ministry of Health (MOH) Malaysia reported cervical cancer as the second leading cause of death after breast cancer among women in year 2003 (Malaysian National Cancer Registry 2003) with an age-standardised incidence of 19.7 per 100,000 population. Its incidence rate by ethnicity was highest among Chinese women, intermediate among the Malays and lowest among Indians. In the third Malaysian National Health Morbidity Survey (NHMS 3) 2006, 43.7% women were reported to have undergone Pap smear examination with highest prevalence among Chinese women (50.5%), other Bumis (Sabahans, Sarawakians) (45.5%), Malays (43.6%), Indian (37.2%) and others (24.7%). The cost of screening and managing pre and invasive cervical cancer (ICC) in Malaysia is calculated to be RM 250 million per year (Wan Puteh et al. 2008).</p>\r\n<p>\r\n	The human papillomavirus (HPV) is considered a public health problem as the virus is sexually transmitted (Kjaer et al. 2001; Lehtinen et al. 2002; Jastreboff &amp; Cymet 2002) and is a major risk factor for cervical cancer development worldwide (Reid &amp; L&ouml;rincz 1996). The International Agency for Research in Cancer (IARC) in 1995 reported that HPV type 16 and 18 are carcinogenic to human. However, more than 100 types of HPV have been discovered (Krejci &amp; Sanchez 2005; Jastreboff &amp; Cymet 2002), of which approximately 15 are oncogenic. Another 30 types of HPV are associated with ano-genital tract infections (Krejci &amp; Sanchez 2005) and type 18 is also found in inverted papilloma and papillary transitional cell carcinoma of the bladder (Chan et al. 1997). Cervical co-infection with multiple types of HPV was postulated to be a predictor of acquisition and persistence of HPV infections and subsequent development of cancer (Rousseau et al. 2001). The prevalence of HPV among normal population (Clifford et al. 2005) shows that it varies greatly between populations, from 1.4% in Spain to 25.6% in Nigeria.</p>\r\n<p>\r\n	HPV types within a species are related, e.g. the Alpha 7 species consists of HPV 18, 39, 45, 59, 68, 70 and a novel c85. Alpha 9 consist of types 16, 31, 33, 35, 52, 58 and 67. The Alpha 10 species consists of the low risk types 6, 11, 13, 44, 55, 74, PcPV and CCPV (De Villiers et al. 2004). HPV infections especially types 16 and 18 (Dillner et al. 1997) are necessary but not sufficient causes of cervical cancer (Yadav et al. 1997; Samoff et al. 2005). Other external cofactors or host factors are required for progression to cancer. There are many risk factors contributing to cervical cancer, these include multiple sexual partners; early sexual debut and activity at a younger age (Nor Hayati 2003); high parity (Hinkula et al. 2004); genital infection e.g. with Chlamydia trachomatis (Hakama et al. 2000; Samoff et al. 2005); herpes simplex virus (Lehtinen et al. 2002); chemical carcinogens (oral contraceptive drugs, cigarette smoking) and low socioeconomic status (Nolte &amp; Walczak 2000). The development of cervical cancer is common in older women even though the HPV infections usually occur in the younger age group (Sellors et al. 2003). In the African region, the mean age of invasive cervical cancer (ICC) among patients was 33.9 years (&plusmn;11.4 years) compared to Southern Europe&nbsp; which is 56.5 years (&plusmn;14.3 years) (Bosch et al. 1995).</p>\r\n<p>\r\n	HPV genotypes have been found to be geographically determined.&nbsp; A previous local study showed that HR HPV genotypes were detected in 95% abnormal smears with HPV 16 being the most prevalent HPV genotype identified (Sharifah et al. 2009). In Europe (Clifford et al. 2005), the prevalence of HPV 16 among women was higher, compared with sub-Saharan Africa, while in Washington, type 16 dominated followed by type 18 (Schwartz et al. 2001), and this follows the general world trend.</p>\r\n<p>\r\n	The distribution of HPV types worldwide showed the same result as Schwartz et al. (2001), except for an earlier study from Indonesia in which HPV type 18 was reported to be the predominant type (Bosch et al. 1995). However, this study was later replicated using a bigger sample, and results showed that HPV positivity was as high as 96% (Schellekens et al. 2004) with types 16 (44%), 18 (39%) and 52 (14%) being the top three dominant types. A study by Chichareon et al. (1998) in Thailand showed that HPV type 16 was most commonly found in squamous cell carcinoma (SCC), followed by HPV types 18, 58, 52, 31, 33 and 39. Bosch and de Sanjosě (2003) also showed that the prevalence of HPV in SCC was around 80% and the five common HPV genotypes found are 16, 18, 45, 31 and 33.&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	From Hong Kong, HPV epidemiology demonstrated a high prevalence of type 52 (Cheung et al. 2008). In Iran, in addition to the predominant types 16/18, there was also a high prevalence of type 33 (Mortazavi et al. 2002). These studies showed that following infection with HPV type 16 and 18, some other types are also predominant and this may vary by regions. This inconsistency raises the question as to whether Malaysia has its own particular HPV distribution that is not in trend with the rest of the world. The<br clear="all" />\r\n	different HPV genotypes may translate into different risks of acquiring cervical cancers according to geographical distributions.</p>\r\n<p>\r\n	With the introduction of the HPV vaccines (quadivalent and bivalent), the protection against cervical cancer is tremendous. The quadrivalent vaccine that protects against types 6, 11, 16 and 18 has the potential to prevent vulval, vaginal and cervical cancers as well as genital warts. There is evidence of cross protection by the quadrivalent vaccine against similar genotypes in the same tree (Bosch et al. 2008). In a cross protection study by Brown (2007), the efficacy of the quadrivalent HPV vaccine against CIN 2, 3 or adenocarcinoma in situ associated with HPV types 6, 11, 16, 18 was 100%, but with increase in numbers of HPV types 31, 33, 34, 45, 51, 52, 56, 58, 59, the efficacy of the vaccine reduces to 38%.</p>\r\n<p>\r\n	Elucidation of HPV genotypes in Malaysian women has not been done on a major scale.&nbsp; This study aims to determine the prevalence and distribution of HPV genotypes in a cohort of Malaysian patients with preinvasive and invasive cervical cancer from Universiti Kebangsaan Malaysia Medical Centre (UKMMC)</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A total of 80 (20 CIN 3, 45 SCC and 15 adenocarcinoma) formalin-fixed, paraffin embedded tumour tissue (PET) blocks were retrieved from the Department of Pathology, Universiti Kebangsaan Malaysia Medical Centre (UKMMC) between 1999 to 2007.&nbsp; Patient consent was obtained through telephone interviews.</p>\r\n<p>\r\n	Five &micro;m thick sections were cut from each case. Gloves and blades were changed during sectioning to avoid cross contamination between samples.DNA was extracted using DNeasy Blood and Tissue Kit (QIAGEN, Germany, Catalog No. 69506). Tissue sections were deparaffinized with xylene and alcohol according to the manufacturer&rsquo;s protocol. Samples were lysed using proteinase K. Lysates were loaded onto DNeasy spin columns. After two washings, pure DNA was eluted in low salt buffer. Presence of DNA was determined by 1% agarose gel electrophoresis. The optical density (OD) measurement was done to determine the purity and concentration of DNA within a ratio of 1.7 to 2.0.&nbsp; The extracted DNA was stored in&nbsp;&nbsp; -20 &deg;C until RT-PCR.</p>\r\n<p>\r\n	HPV genotyping was carried out using SACACE HPV High Risk Typing Real-TM kit (SACACE, Italy, Catalog No. TV26-100FRT). This kit is an in-vitro Real Time amplification test for qualitative detection and genotyping of 12 HR-HPV genotypes (16, 18, 31, 33, 35, 39, 45, 51, 52, 56, 58 and 59). It contains four PCR-mix tubes; each of which contain primers directed against regions of three HPV genotypes with the &beta;-Globin gene as an internal control.Multiplex amplification reaction was performed in a volume of 13 &mu;l containing 8 &mu;l of reaction mix (PCR-mix-1, PCR-buffer FRT, Hot Start DNA Polymerase) and 5 &mu;l of DNA sample. Each reaction was run for 45 cycles, under the following conditions: 15min at 95&ordm;C, 20 sec at 95&ordm;C and 60 sec at 60&ordm;C. Data obtained from Real-Time PCR is then keyed-in to a software (Microsoft&reg; Excel HPV Typing Real-Time MX Results Matrix.xls) provided by the SACACE kit, and interpreted.&nbsp; Statistical analysis was performed using Microsoft&reg; Excel 2007.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	A total number of 80 PET samples were analyzed (20 CIN 3, 45 SCC and 15 adenocarcinoma). The mean age of patients was 52 years, ranging from 30 to 80 years (Table 1). The highest number of HPV positive cases were from the 50-59 year age group (32.5%) with the Chinese showing the highest prevalence (66.3%) followed by Malay (25%) and Indian (5%) (Table 2).</p>\r\n<div>\r\n	<p>\r\n		Of the 80 samples of preinvasive and invasive cervical cancer (ICC), 92.6% showed presence of HPV genotypes. Twelve different HPV genotypes were detected i.e 16, 18, 31, 33, 35, 39, 45, 52, 58, 59, 6 and 11. Among the CIN 3 cases, HPV 16 was the most prevalent (60%), followed by HPV 33 (35.0%), HPV 45 (25.0%), HPV 18 (15.0%) and HPV 39 (15.0%). HPV 16 (80.0%) was the most prevalent genotype in SCC of the cervix, followed by HPV 45 (54.5%), HPV 33 (31.1%), and HPV 52 (26.7%). The prevalent HPV genotypes in adenocarcinoma of the cervix were HPV 16 (73.3%), 18 (46.7%), 33 (20.0%), 39 (6.7%) and 59 (6.7%) (Table 3). The five most prevalent HPV genotypes in 80 samples of preinvasive and ICC were HPV 16 (73.8%), 33 (30.0%), 18 (22.5%), 39 (16.3%) and 52 (16.3%) (Figure 1). The five most prevalent HPV genotypes in ICC were 16 (78.3%), 33 (28.3%), 18 (25%), 52 (20%) and 39 (16.7%) (Figure 2).</p>\r\n	<p>\r\n		Multiple infections of at least two HPV genotypes were noted in 53.8% of the cases. 38.8% showed presence of a single genotype while 31.3% had two genotypes, 12.5% had three genotypes, 5% had four genotypes and 5% had five genotypes. Multiple HPV genotypes were more prevalent (53.8%) than single HPV genotype (38.8%) (Table 4).</p>\r\n	<p>\r\n		<strong>DISCUSSION</strong></p>\r\n	<p>\r\n		The higher prevalence of HPV in Chinese ethnicity among sampled patients diagnosed to have cervical cancer in this study was consistent with results from the Malaysian Cancer Registry (2003) which reported that the incidence are highest among the Chinese with 28.8 per 100,000, followed by Indians (22.4) and Malays (10.4). Even though samples were from UKMMC which is a public teaching hospital, with a high number of Malay patients, results showed that the highest percentage of cervical cancer was found in the Chinese patients (66.3%). Malays (25%) and Indians (5.0%) showed lower prevalence of cervical cancer in this study. This result reflects the findings of cervical cancer incidence at the National level. In a Muslim country, and coming from a Malay culture that strictly prohibits cohabitation or sex outside wedlock may have a role in the reduction of risky sexual behaviours among its youths. Findings from the National Health Morbidity Survey 3 (NHMS 3) 2006 showed that an estimated proportion of those who had genital discharge/ulcers and who subsequently came forward for treatment were Chinese (49.9%). The prevalence profile of those who had genital discharge/ulcer was highest among the Indians (3.5%) followed by Chinese (2.9%) and Malays (1.7%) (NHMS 3 2006).</p>\r\n	<p>\r\n		Results of this study did not show any association between age and HPV positivity, and numbers of HPV types. In a study by Tang (2002), age was found to be inversely associated with HPV infections. Young females were more likely to harbour HPV, and oncogenic HPVs were found in patients below the age of 61 years old. Women in this study were from the middle age to elderly age groups with a mean age at 52 &plusmn; 12.2 years. In this study, 28.8% cases aged 60 and above, presented in the later stages of cancer. The Durex sexual survey (1999) and Sexual and Reproductive Health of Adolescents and Youths in Malaysia (2005), showed that sexual debut for women in this country was highest among the 17-18 year old. During adolescence, the knowledge of early sex and its&rsquo; risk management is poor. NHMS 3 (2006) showed that the knowledge on symptoms of sexually transmitted illness was low, (50.9-53.8%) especially among the younger age group (below 19 years old) and the extreme age (above 70 years old). Findings from the NHMS 3 (2006) showed that females had their first sexual intercourse at the age of 22.8 years and initiation of sex does not vary much by ethnicity. However, this is an understatement as young adolescents do not declare sexual initiations to parents and guardians openly. This sexual novelty of a young woman with usually a more mature and experienced partner, puts her at risk of acquiring the infection through sexual intercourses and transmission to other partners later on. Data on the HPV prevalence and types of HPV among men is not readily available, and the cost effectiveness of HPV vaccination among men are still debated. The development of infection takes time, sometimes up to decades (Sankaranarayanan et al. 2001) with most clearing the infection within one year (Sellors et al. 2003). Women usually become infected when they are younger (Rousseau et al. 2001; Clifford et al. 2005), and some infections do not clear and progresses into dysplasia and frank cancer. HPV 16 infection (new infections) was associated with an increased risk of acquisition of other HPV types (Rousseau et al. 2001) and increases the risk of ICC (Dillner et al. 1997). By this time the women are usually in their older age group. From this study, we noted the presence of cancer among &lsquo;relatively younger&rsquo; women i.e. less than 52 years old (mean age of cases in this study). Multiple infections play a role in oncogenesis and reduce cellular resistance, thus accelerating progression to cancer. In the 30-39 year age group, 41.7% had developed CIN 3, 75% had infection with type 16, 16.7% had infection with type 18 and 33.3% showed type 33. A high percentage of multiple infections was also noted in the younger age group.</p>\r\n	<p>\r\n		Vinh-Hung et al. (2007) reported that SCC accounted for two thirds of the incidence of cervical cancer in the United States. Our results showed HPV types 16, 18, 33 and multiple infections dominated in SCC. Type 18 was also found at a high percentage in adenocarcinoma (46.7%). Both types 16 and 33 are from the Alpha 9 species, while type 18 is from the Alpha 7 species. Our results also showed that HPV positivity in ICC was highest in SCC (95.6%).</p>\r\n	<p>\r\n		Lehtinen et al. (2002) in their study among SCC, showed that HPV type 16, 18 and 33 were associated with ICC especially types 33 and 16. According to Tang (2002) oncogenic HPV types are less infectious but cause more persistent infections than their lower risk counterparts, thus, repeated exposures are needed for infection to occur.</p>\r\n	<p>\r\n		Clifford et al. (2005) reported that HPV 16 was twice as frequent as any other high-risk type in all regions except the sub-Saharan Africa where HPV 35 was equally common. The next common types were type 33 and 56 in Asia, 58 in South America and 31 in Europe. In our patients with CIN 3 and ICC, the prevalence of HPV 16 was found to be as high as 73.8%.</p>\r\n	<p>\r\n		These current findings showed that types 16 and 18 play a dominant role in cervical carcinogenesis, both globally as well as in Malaysia.&nbsp; However, the high prevalence of HPV type 33 is a deviation against the normal trend except in Iran (Mortazavi et al. 2002), where the common HPV that predominate are types 16, 18 followed by 33. In the Iranian study, the prevalence of HPV in ICC was 85.5% which is nearly as high as the findings from our study which was 93.3%. Banura et al. (2008) reported that among the 12-24 year old sexually active women, the prevalence of HPV genotypes by decreasing order are 52, 51, 18 and 16. Other studies have also reported similar results (Clifford et al. 2005) in which type 33 and 56 are common among Asian women.</p>\r\n	<p>\r\n		HPV 16/18 vaccines are estimated to provide about 67% protection against ICC in Asia (Bao et al. 2007). The difference in the prevalence of HPV types by different regions is shown in a mass study of women by Bao et al. (2007); by decreasing frequencies: HPV 16, 18, 58, 52, 33 in East Asia (China, Hong Kong, Taiwan, Korea, Japan); HPV 45, 52, 58 in South-East Asia; and HPV 45, 33 and 35 in South Central Asia (Bao et al. 2007). The difference in the HPV prevalence need to be considered before mass vaccination of women is carried out, if the prevalence of HPV is not within the bivalent or quadrivalent vaccine efficacy coverage. However, results from our HPV genotyping suggests that a vaccine that covers both HPV types 16 and 18 will be able to cover 54.9% of this disease.</p>\r\n	<p>\r\n		From our study of preinvasive and ICC, HPV type 16 was the most prevalent type detected (73.8%), followed by type 33 (30%) and type 18 (22.5%). With the different variations seen and different infectivity rates reported from various countries and regions, it is very important that local genotypes be properly determined and elucidated. The results of our study, reflect the prevalence and distribution of HPV genotypes in a cohort of Malaysian patients with preinvasive and ICC.</p>\r\n	<p>\r\n		<strong>CONCLUSION</strong></p>\r\n	<p>\r\n		HPV infection is very high among high grade preinvasive lesions and ICC (92.5%) in Malaysian women. The five most prevalent HPV types are HPV 16, 33, 18, 39 and 52. Multiple HPV infections (53.8%) are relatively common compared to single infections. If the same HPV types represent the country&rsquo;s HPV infection trends among local high risk or cancer inflicted women, a vaccine that prevents infections from HPV types 16 and 18 will protect as high as 54.9% of women in this society.</p>\r\n</div>\r\n', 1),
(452, 449, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	The incidence of premature deaths in Ma&shy;laysia involves 33,623 patients admitted into government hospitals for acute coro&shy;nary syndromes (Malaysian Clinical Prac&shy;tice Guidelines on UA/NSTEMI 2002) with a mortality rate of 15.7% total death in 2006. Coronary artery bypass surgery has increased the survival rate of the individu&shy;als suffering from coronary heart disease (CHD). According to the Ministry of Health (MOH), coronary heart disease is one major cause of premature deaths in Ma&shy;laysia, re&shy;sulting in significant psychosocial and eco&shy;nomic implications in the country (NCVD ACS Registry 1st Annual Report 2006).</p>\r\n<p>\r\n	Through research and improved treat&shy;ment modalities, the survival rate for those afflicted with CHD has improved but the prevalence of the disease re&shy;mains the same (Rosamond et al. 2007). There is statistical evidence confirming that coronary artery bypass surgery im&shy;proves the survival rate of patients with acute or chronic coronary ar&shy;tery dis&shy;eases. CABG surgery has been touted as the gold standard of treatment for pa&shy;tients with CHD. Being one of the most popular treatments to revascularize blocked coronary arteries, CABG surgery saves lives and reduces angina to im&shy;prove the quality of life (QOL) (Mohamed et al. 2009; Khoo et al. 1997). The multi&shy;dimensional aspects of health re&shy;lated QOL requires a multivariate analysis of the physical, psychological, and emo&shy;tional well-being to evaluate QOL post interventions. The impact of coro&shy;nary artery intervention on the life function&shy;ing post intervention is more important to the patient than being a survivor (Graham et al. 2006; Najafi et al. 2008).&nbsp; The patient must evaluate their expectations of post operative recovery of CABG. A previous study reported that in most patients, CABG improved their QOL following sur&shy;gery (Barbareschi et al. 2009). However, the recovery period for each individual varies according to their specific level of QOL which is an indicator within a specified period of time. This is not seen with a typical rehabilitation phase (Ballan &amp; Lee 2007; Merkouris et al. 2009).</p>\r\n<p>\r\n	The Toronto QOL researcher&#39;s definition of QOL included the va&shy;riables of the indi&shy;vidual&#39;s physical, psycho&shy;logical, and spiri&shy;tual functioning, their con&shy;nections with their environment, and oppor&shy;tunities for main&shy;taining and enhancing skills (Revicki 1989). These variables can also be seen in the World Health Organization&#39;s definition of health and health promotion (WHO 1998). Both entities agreed that the definition of QOL is multifaceted and a working definition was drafted to support the area of this study.</p>\r\n<p>\r\n	This study should provide nurses with the knowledge of a patient&#39;s expectations of QOL and an understanding of the value of the QOL domains as valued by the patient. Little is known about percep&shy;tion of QOL for patients who have under&shy;gone CABG surgery when we as nurses are actively involved in patient care. This research was expected to measure the QOL in individuals who have expe&shy;rienced CABG by assessing the patient&rsquo;s perception of functioning within the physical and mental domains of the Medical Out&shy;comes Short Form 36 (SF-36) (Ware et al. 1994). The main objec&shy;tive of this study was to examine the per&shy;ception of QOL amongst post CABG pa&shy;tients at the National Heart Institute (NHI).</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	This cross-sectional descriptive study was conducted to examine the percep&shy;tion of QOL amongst post CABG patients at the National Heart Institute (NHI). Data collection was carried out after approval from the Institutional ethicsand research committee (project code FF-032-2008). The Medical Outcomes Short Form 36 (SF-36) as general health status con&shy;sisted of 36 multiple-choice questions sorted into two components:&nbsp; physical component summary (PCS) and mental component summary (MCS) (Ware et al. 1994). It comprised of eight assessment scales: 1) physical function&shy;ing measures the limitations in physical activity due to health problems; 2) social functioning measures the limitation of social activities because of physical and emotional problems; 3) role physical items identify limitations in usual role activities because of physical health problems; 4) bodily pain items to assess presence of pain and limitations due to pain; 5) gen&shy;eral medical health include self evaluation of health; 6) mental health items measures psychological distress and well-being; 7) role limitation measures the limitations in usual role activities because of emotional problems; and 8) vitality items to assess energy and fatigue levels of the respon&shy;dents. These domains were used to measure the QOL in post CABG patients at the NHI. The use of the SF-36 scales has been reported in 14 studies researching vari&shy;ous disease processes and social condi&shy;tions using random samples in both the United States and the United Kingdom (Ware et al. 1994). Socio-demographic data in&shy;cluding gender, age, race, marital status, education level, occupation, in&shy;come, du&shy;ration of post CABG, physical exercises and duration of the physical exercises were recorded in the study. A conceptual framework of the study is de&shy;picted in Figure 1.</p>\r\n<p>\r\n	The inclusion criteria of respondents were patients having CABG surgery in NHI who on agreed to sign a written con&shy;sent. A total of 69 post CABG patients at the NHI, were recruited in this study. Statistical Pack&shy;age for Social Science (SPSS) 12.1 for Windows was used for statistical ana&shy;lyses. Descriptive analyses include fre&shy;quency of the respondents&rsquo; socio-de&shy;mo&shy;graphic, mean and standard deviation on the score of QOL. Inferential analysis such as t-test and Analysis of Variance (ANOVA) test were used to compare the relationship between socio- demographic data and QOL of the post CABG respon&shy;dents.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Six nine participants were studied. Res&shy;pondent&rsquo;s socio-demographic profiles including gender, age, race, marital sta&shy;tus, education level, occupation, income, physical activities and duration of physi&shy;cal activities were depicted in Table 1.</p>\r\n<p>\r\n	Perceptions of QOL amongst CABG patients were calculated for all compo&shy;nents and the two summary scores for physical health and mental health is pre&shy;sented in Table 2. The mean for each SF-36 subscales, physical domains and mental domains were classified into level of poor QOL with the score of 0-49 and good level of QOL with the score of 50-100. The find&shy;ings showed that post CABG respon&shy;dents possessed a good level of QOL with an average score of 57.80 &plusmn; 25.145. In Table 2, the MCS score (64.02 &plusmn; 25.470) was found to be higher than PCS score (51.58 &plusmn; 24.881). Results of this study it revealed that post CABG patients in NHI were reported to have good QOL.</p>\r\n<p>\r\n	Student t-test was performed to com&shy;pare the mean of SF-36 subscale and socio-demographic profiles. There were signifi&shy;cant differences between the role func&shy;tioning of sub-components of physical functioning domain with gender (t= .286, p &lt;0.05). Male respondents have higher score in role physical sub-components (56.91&plusmn;47.119) compared to female (29.55 &plusmn;22.001). Gender however, was not significantly different with other sub-domains of SF-36 items (Table 3).&nbsp;</p>\r\n<p>\r\n	Duration of physical exercises of more than 20 minutes have positive effects on both the physical component (t=2.738, p &lt; 0.05) and mental component (t=7.326, p &lt; 0.05) (Table 4). Marital status of the respondents was found not signifi&shy;cantly different with other sub-domains of SF-36 items.</p>\r\n<p>\r\n	Analysis of Variance (ANOVA) test was used to compare the mean of SF-36 subscale and the socio-demographic pro&shy;files. Ethnicity, age groups and post CABG period showed no significant differ&shy;ence with sub-domains of SF-36 items.</p>\r\n<p>\r\n	The education level showed significant differ&shy;ence with the general health of physi&shy;cal component domain (F= 4.962, p &lt;0.05). The findings showed that the highest score was for tertiary education (60.45&plusmn;9.28), fol&shy;lowed by primary education (43.17 &plusmn; 17.145) and secondary education (41.61 &plusmn; 16.835). All other-domains of SF-36 items were found to be not significant with the education level (Table 5).</p>\r\n<p>\r\n	Respondents&rsquo; income showed signifi&shy;cant difference with the role physical (F= 5.300, p &lt;0.05) and physical component summary (F= 3.505, p &lt;0.05) of the SF-36 items. An income of RM1001 to RM 3500 score highest (86.36 &plusmn; 32.333), compared with an in&shy;come of RM 3501 (52.27&plusmn;43.952) and less than RM1000 (38.30&plusmn;46.582). There were significant differences with the role emotional (F=3.434, p&lt;0.05), mental health (F=3.835, p&lt;0.05) and mental compo&shy;nent sum&shy;mary (F=4.272, p&lt;0.05). The respon&shy;dents&rsquo; income of RM 1001 to RM 3500 was found to have the highest score of role emo&shy;tional (90.91&plusmn;30.151), mental health (88.00&plusmn;14.311), and men&shy;tal component summary (83.59&plusmn;20.308) (Ta&shy;ble 6). However, other sub-domains did not show significant difference as de&shy;picted in Table 6.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	The results of this study showed that post CABG patients in NHI have good QOL. The MCS scores seemed to have better rating than the PCS scores of the SF-36. This result was in accor&shy;dance to earlier findings which reported on the importance of having a structured multi-disciplinary reha&shy;bilitation program with focus on emo&shy;tional support, information and education needs (Merkouris et al. 2009; J&auml;rvinen et al. 2003). A previous study revealed that some patients may present with physical health problems which were re&shy;flected in their health related to QOL five to fifteen years post CABG (Herlitz et al. 2009:&nbsp; Rantanen et al. 2009). In contrast, another study by Peric et al. (2010) about predictive fac&shy;tors for decreasing QOL six months post CABG were female gender, diabetes mellitus, low ejection fraction, and the presence of postoperative com&shy;plications. This could be attributed to different lifestyles among the two popula&shy;tions.</p>\r\n<p>\r\n	This study reported gender-related dif&shy;ferences in QOL with the role physical functioning among the post CABG res&shy;pondents. There were signifi&shy;cant differ&shy;ences between the role func&shy;tioning sub-components of physical func&shy;tioning do&shy;main with gender. Male res&shy;pondents have higher score in role physical sub-components, compared to female. Ac&shy;cording to Phillips et al. (2003), even af&shy;ter adjusting for known risk factors for compromised QOL, women do not show quality benefits of CABG surgery that men do. However, Koch et al. (2007) empha&shy;sized that attention related to preoperative conditions, such as conges&shy;tive heart failure, anemia, diabetes, and advanced age, were indicative of greater risk in both women and men for lower survival post CABG surgery. Most im&shy;portantly the efforts to reduce or modify such disease prevalence earlier in women, may allow longer survival after surgical intervention. Differ&shy;ences in post&shy;operative survival between women and men were related to gender differences in the distribution of preoperative risk factors (Koch et al. 2003; Peters 2001).</p>\r\n<p>\r\n	Education levels showed sig&shy;nificant difference with the general health of physical component domains of SF-36 items. Conversely, respondents with ter&shy;tiary education possessed the highest score followed by primary education and lastly secondary education. A study by LeGrand et al. (2006) confirmed that most patients report an improvement in their QOL following coronary ar&shy;tery by&shy;pass surgery. However, the re&shy;covery pe&shy;riod for each individual varies in congru&shy;ent with the above mentioned study.</p>\r\n<p>\r\n	Consequently, our results showed that physical exercises and its duration have signifi&shy;cant impacts on the QOL as re&shy;ported in this study. Exercises of more than 20 minutes have positive effects on all physical and mental component do&shy;mains except for the physical functioning sub-domain. Physical exercises per&shy;formed by CABG patients enhanced mental health as it entails mental en&shy;gagements which thereby reduce the stress level of the patients. Generally, the</p>\r\n<p>\r\n	QOL score depend on the per&shy;formance of physical exercises especially for the CABG patients, although, other studies have stated that the survival and QOL benefits should not be re&shy;garded as a substitute for revascularization (Markou et al. 2008; Najafi et al. 2008). A previous study by J&auml;rvinen et al. (2003) reported that elderly patients not only have higher mortality and morbidity but also derive less benefit from CABG on certain as&shy;pects of QOL.</p>\r\n<p>\r\n	The NHI pro&shy;vides cardiac rehabilitation program to pre and post CABG patients whilst hospi&shy;talization. Subsequently, the advance&shy;ment of technology and mass media deployed at the NHI has created awareness to&shy;wards CABG patients with regards their con&shy;dition and continuity of care. Once CHD pa&shy;tients are discharged, they are closely monitored and put on regular follow up. They are contacted via telephone by the car&shy;diac clinic staff if they fail to turn up on the appointment date. Similar findings were reported regarding the importance of having supportive groups which are required to monitor the progression in the physical and mental domains of CHD patients (Bergh et al. 2007).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The post CABG patients had good QOLS post operatively. They were able to make comprehensive lifestyle changes through frequent physical exercises to accommo&shy;date their needs. This could be the result of pre and post CABG counseling and health education. This study confirmed the importance of cardiac rehabilitation program as supporting evidence in the improvement of quality of life amongst CABG patients.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGMENTS</strong></p>\r\n<p>\r\n	The authors would like to express their greatest gratitude to UKM fundamental grant, NHI Manage&shy;ment for granting us the permission to collect the data in this study. The authors sincerely thank the Institution for sanc&shy;tioning the grant FF-032-2008. In addi&shy;tion, the authors are thankful to Assoc Prof. Dr. Srijit Das, and Dr. Tan Aik Kah for their critical comments on the paper.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(453, 450, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Facial nerve schwannoma (FNS) is a rare slow growing benign tumour. Schwannomas arise from the Schwann cell of the neurilemma and can involve the cranial nerve, spinal nerve and sym&shy;pathetic nerve. In 60% of cases, schwannoma usually arises from the cranial nerves, commonly of the sensory type (Sarma et al. 2002). Of all the possi&shy;ble cranial nerves involved, vestibular schwannomas are the most common. In a reported series of 46 patients the commonest nonvestibular schwannomas are trigeminal (56.5%), followed by facial schwannomas (15.2%), glossopharyn&shy;geal nerve (15.2%), vagus and spinal accessory nerve (4.4% patients), hypog&shy;lossal schwannomas (6.5%) and oculo&shy;motor (2.2%). Involvement of other cranial nerves such as trochlear and ab&shy;ducens nerves are rare.</p>\r\n<p>\r\n	Facial nerve schwannoma can involve any segment of the nerve where each has a unique presentation. It is the com&shy;monest tumour among primary facial nerve tumours. This case series review aims to provide data on the clinical pres&shy;entation of FNS and its management.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A retrospective analysis of 6 consecutive cases of FNS managed between 1998 and 2008 was conducted. All these pa&shy;tients had histological confirmation of schwannoma from their surgical speci&shy;mens. The location of the tumor was de&shy;termined by imaging pre-operatively and confirmed intra-operatively. Tumours arising within the tympanic and mastoid segment were grouped as intratemporal tumours. The facial nerve function was evaluated with the House-Brackmann facial nerve grading system.&nbsp; The post&shy;operative evaluation for recurrence was performed either clinically or in patients with intracanalicular FNS, and evaluation was by magnetic resonance imaging.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Six patients (3 females, 3 males) with an average age of 42 years (age range 19 to 66 years) were included in this study (Table 1). The mean follow up period was 2 years. The FNS originated in the internal auditory canal (IAC) (2 cases, 33%), intratemporal (3 cases, 50%) and intra&shy;parotid (1 case, 16.6%) segments of the facial nerve. The commonest initial pre&shy;senting symptom was reduced hear&shy;ing and tinnitus (5 out of 6 cases). Pro&shy;gres&shy;sive facial nerve palsy occurred in 3 pa&shy;tients during the course of the disease (2 with intratemporal FNS and 1 with IAC FNS). The patient with intraparotid FNS only presented with parotid swelling and did not have facial nerve (FN) palsy or hearing loss. In this current series we observed that tu&shy;mours which involved the tympanic seg&shy;ment would initially present with tinnitus, ear blockage and conductive hearing loss followed by FN palsy. How&shy;ever, there was one patient who devel&shy;oped progressive sensori&shy;neural hearing loss which could be explained by bony ero&shy;sion of the basal turn of the cochlea by the tumour.</p>\r\n<p>\r\n	The three patients with intratemporal FNS had successful tumour excision via transmastoid approach. Otoscopic ex&shy;amination revealed a mass medial to the tympanic membrane (Figure 1). Intra&shy;operative findings revealed FNS involv&shy;ing the tympanic segment in these three patients. The continuity of the facial nerve was preserved in 2 patients. The other patient had tumour excision in&shy;cluding part of the facial nerve which was then repaired by sural nerve cable graft&shy;ing. The facial nerve had to be resected as it was adherent to the tumour and could not be separated.</p>\r\n<p>\r\n	The patient with intraparotid FNS had complete removal of the tumour following superficial parotidectomy. Pre-operative imaging showed features of a benign pa&shy;rotid tu&shy;mour. Fine needle aspiration cy&shy;tology was inconclusive. Intra-operative findings revealed a partially cystic tumour arising from the facial nerve trunk. In this case the facial nerve was sacrificed and end-to-end facial nerve anastomoses were performed.</p>\r\n<p>\r\n	Both patients with FNS involving the IAC presented with hearing loss and tin&shy;nitus. One of them is a cook who pre&shy;sented late with complaint of dysacusia despite having had facial nerve palsy for nine months. He also had profound sen&shy;sori&shy;neural hearing loss. Intra-operative find&shy;ings showed a large tumour at the IAC involving the geniculate ganglion ex&shy;tending superiorly into the middle cranial fossa which corresponded to the pre-operative imaging studies (Figures 2 and 3). The tumour was removed via a mid&shy;dle cranial fossa approach.</p>\r\n<p>\r\n	The other patient with IAC FNS pre&shy;sented with a 2 year history of hearing loss and tinnitus with recent onset of im&shy;bal&shy;ance. Imaging studies showed tumour arising from the IAC with extension into the cerebellopontine angle. A pre-opera&shy;tive diagnosis of vestibular schwannoma was made. Intra-operative findings re&shy;vealed the tumour originated from the facial nerve. The tumour was success&shy;fully excised via posterior fossa craniot&shy;omy approach. In both these patients, the FNs were sacrificed and could not be anastomosed due to a short proximal stump.</p>\r\n<p>\r\n	Patients who had preservation of thefa&shy;cial nerve continuity had the same func&shy;tional grading pre- and post-operatively. Two patients who had re-anastomoses or cable grafting had deterioration of the facial nerve function from normal to grade III and IV respectively. Another two patients with IAC FNS whose facial nerve could not be grafted had post-operative facial nerve grade V (Table 1).</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Primary facial nerve tumor is rare with an incidence of 1:23,000 ear cases (Sham&shy;baugh et al. 1969). The commonest pri&shy;mary facial nerve tumor is schwannoma. Falcioni et al. (2003) had reported a se&shy;ries of 28 primary facial nerve tumour in which 18 were schwannomas, 6 were hemangiomas, 2 were meningiomas and 2 were neurofibromas. FNS may arise from any segment of the facial nerve from the cerebellopontine angle (CPA) to the peripheral branch in the parotid gland. Multiple-segment localization can also occur. Sherman et al. (2002) re&shy;ported that the most common segment involved is the labyrinthine (43.5%), fol&shy;lowed by tympanic (42.8%), vertical (36.7%), canalicular (24.3%), cerebello&shy;pontine angle (17.8%), peripheral (15%), chorda tympani (1.2%) and nerve to sta&shy;pedius (0.9%) in his literature review of 428 cases.</p>\r\n<p>\r\n	The presenting symptoms of FNS may mimic any tumour that can arise at the same origin. Patients usually present late due to the non-specific symptoms. Sus&shy;picion is raised pre-operatively when pa&shy;tients present with progressive or re&shy;laps&shy;ing facial nerve palsy. In reality, many pa&shy;tients present with hearing loss and tinni&shy;tus as observed in this study. Sherman et al. (2002) in his meta-analy&shy;sis of cases of FNS had shown that facial weakness was present in 63.2% of pa&shy;tients, hearing loss in 50.6% and tinnitus in 20.6%. Other symptoms including otalgia, otorrhoea and loss of taste were seen in less than 10%. On the other hand, his personal case se&shy;ries showed hearing loss as the most common pre&shy;senting symptom, similar to the current study. In addition, many FNS cases are diagnosed intra-operatively and confirmed by histology of surgical speci&shy;mens. The presence of facial nerve palsy also gives no indication to the exact site of origin as observed in our series as well. A pre&shy;vious study suggested that facial nerve palsy is more prevalent in intratemporal tumours when compared to extratem&shy;poral tumours (Park et al. 2007).</p>\r\n<p>\r\n	Extratemporal FNS is the most un&shy;com&shy;mon site to be involved. Interestingly in this series there was one patient who pre&shy;sented with 3 years history of pro&shy;gressive painless parotid tumor without facial nerve palsy. The diagnosis was only suspected intra-operatively. A retros&shy;pective study of 8 cases of intraparotid schwannoma by Caughey et al. (2004) showed that only 37.5% of them pre&shy;sented with facial nerve palsy. Therefore, it is often difficult to diffe&shy;rentiate FNS from other common benign parotid tu&shy;mours.</p>\r\n<p>\r\n	It is also difficult to differentiate intraca&shy;nalicular FNS from vestibular schwan&shy;noma (VS) especially when the only pre&shy;senting symptoms are hearing loss and tinnitus. Park et al. (2007) in a series of 11 cases of intranalicular schwannomas, had reported 2 (11%) cases of FNS. In both cases, the diag&shy;noses were made intra-operatively. There is no gold stan&shy;dard objective pre-opera&shy;tive assessment which can confirm the diagnosis for bet&shy;ter counselling of the patient. Previous studies recommend that there is post&shy;erior displacement of the tumour in FNS relative to the IAC in magnetic resonance imaging (MRI) (T1 weighted images post gadolinium). How&shy;ever, when the FNS fills up the entire di&shy;ameter of the IAC it can no longer be differentiated from VS (Caughey 2004, J&auml;ger &amp; Reiser 2001). On the other hand, the presenting symptom of facial nerve palsy more likely supports the diagnosis of FNS as shown in one of our cases be&shy;cause facial nerve palsy in VS is ex&shy;tremely rare (O&rsquo;Donoghue 1989).</p>\r\n<br clear="all" />\r\n<p>\r\n	The recommended radiological imaging in cases of suspected FNS is CT or MRI. The radiological findings of FNS are sim&shy;ilar to schwannoma of other nerves. CT scan will show an enhancing soft tissue mass with lack of bony destruction due to its benign nature. Bone algorithm will demonstrate an expansile bony lesion with lytic changes or remodeling of bone. This imaging is most useful in showing the extension of intratemporal FNS (Fig&shy;ures 2 and 3).</p>\r\n<p>\r\n	On the other hand, MRI gives the best soft tissue quality and will show a mildly hypo or isointense lesion relative to the brain in T1-weighted images. On T2-weighted images, the tumor is heteroge&shy;neously hyperintense which will further enhance with gadolinium (Ginsberg &amp; DeMonte 1999). In cases of intracanali&shy;cular FNS, combined modality of CT and MRI is rec&shy;ommended to differentiate be&shy;tween FNS and VS (Martin et al. 1992). MRI findings of FNS may show presence of labyrin&shy;thine tail which represents tu&shy;mour exten&shy;sion into the labyrinthine canal (Ramina et al. 2008). High resolution CT findings of pressure erosion at the antero-supe&shy;rior margin of the IAC and/or subtle bone erosion of the first segment of the fallo&shy;pian canal are suggestive of FNS. How&shy;ever, these findings are irrelevant in large intracanalicular tumours (Caughey 2004, J&auml;ger &amp; Reiser 2001).</p>\r\n<p>\r\n	The mainstay treatment for FNS is sur&shy;gical excision. The aim of surgery in FNS is complete tumor excision with preser&shy;vation of the facial nerve and hearing function. The timing and method of sur&shy;gery depend on the presenting facial nerve function, location and size of the tumor and hearing status. As majority of the cases of FNS is diagnosed intra-ope&shy;ratively, the decision on preservation of facial nerve can be only determined on table. Therefore, pre-operative patient counselling regarding facial nerve func&shy;tion risk is of utmost importance in all cases. In most symptomatic cases, sur&shy;gical resection with sacrifice of the in&shy;volved facial nerve segment is often ne&shy;cessary. Some authors advocate that a facial nerve palsy of House Brackmann grade III or worse should encourage sa&shy;crifice of the facial nerve followed by end to end anastomosis or grafting (Chung et al. 2004).</p>\r\n<p>\r\n	Patients with minimal facial or no facial dysfunction poses the greatest dilemma to the surgeon. This is because surgical treatment would inevitably lead to some degree of facial paralysis. However, some authors believe that patients with minimal facial dysfunction of shortest duration pre-operatively will have better facial nerve function outcome after surgery (Yamaki et al. 1998). They argue that in these cases the shortest distance for anastomosis is still achievable.&nbsp; When the tumour is large there is increased risk of compression of important structures such as the brains&shy;tem and the surgery becomes technically challenging. A longer segment of the nerve has to be resected, that end to end anastomosis is not possible and inability to graft the re&shy;sected segment is due to short proximal nerve stump especially in intracranial FNS. Although surgical resec&shy;tion is the primary mode of treatment, other treat&shy;ment options can be consi&shy;dered. Liu &amp; Fagan (2001) had conserva&shy;tively ma&shy;naged 8 out of 10 patients with tumors confined to the intracanalicular and cere&shy;bellopontine angle. The patients had serial imaging for tumour monitoring. They showed that 80% of the patients maintained their facial nerve function for up to 10 years with no significant tumour growth on serial imaging. Stereotactic radiosurgery has been considered for management of FNS in a small number of patients. It is a viable option for those pa&shy;tients who are medically unfit for sur&shy;gery (Sherman et al. 2002). A recent ar&shy;ticle had highlighted the role of stereo&shy;tactic radiotherapy in FNS (Nishioka et al. 2009). In four patients with FNS, they had shown that the tumour size decreased or unchanged after a median total dose of 59 Gray. The facial nerve function of these patients also improved or remained the same post treatment with a mean follow up of 67.3 months. They con&shy;cluded that stereotactic radiotherapy is as effective as stereotactic radiosurgery and may prove to be more efficacious in preserving neu&shy;rological function. How&shy;ever, a larger follow up study needs to be performed to prove this claim.</p>\r\n<p>\r\n	Facial nerve function is inevitably at risk in surgical resection of FNS. However, as it is often an intra-operative diagnosis, surgical consent of facial nerve palsy should always be sought. Park et al. (2007) had shown in a series of intra&shy;ca&shy;nalicular schwannomas, the possibility of facial nerve schwannoma was 11%. When the facial nerve integrity has to be sacrificed, end to end anastomosis or ca&shy;ble graft are the preferred choices for best prognosis of facial nerve function. Liu &amp; Fagan (2001)in their review noted that the best result can be obtained with cable grafting is House-Brackmann grade III. Preservation of facial nerve is possible in some cases by stripping sur&shy;gery, that is, microdissection at the junc&shy;tion of the nerve and tumour capsule or surgical decompression of the tumour especially at the cerebellopontine angle. This approach is advocated in those with good pre-operative facial nerve function (Kim et al. 2009).</p>\r\n<p>\r\n	The principles of surgical approach de&shy;pend mainly on the tumour origin, tumour extension and hearing function. Intra&shy;temporal FNS are approached via the mastoid with hearing preservation. Tu&shy;mours involving the labyrinthine segment are removed via middle cranial fossa ap&shy;proach with hearing preservation or translabyrinthine approach in those with non-serviceable hearing. In large tu&shy;mours involving the cerebellopontine an&shy;gle the posterior cranial fossa approach is a viable option.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	The presenting symptoms of FNS are commonly hearing loss and progressive facial nerve palsy which are non specific. Pre-operative diagnosis still remains a diagnostic dilemma especially when the facial nerve function is normal. Patients should always be counselled regarding facial nerve paralysis before any ear sur&shy;gery because the diagnosis of FNS is often made intra-operatively. The integr&shy;ity of the facial nerve should be pre&shy;served whenever possible. If part of the nerve is sacrificed, end to end anatomo&shy;sis or ca&shy;ble grafting gives the best out&shy;come for facial nerve function.</p>\r\n', 1),
(454, 451, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Ranibizumab (Lucentis<sup>TM</sup>, Genentech Inc, San Francisco, US) is a humanized antigen-binding fragment designed to inhibit all iso&shy;forms and active degradation products of vascular endothelial growth factor A (VEGF-A) (Gaudreault et al. 2007). Ranibizumab&nbsp; (Rosenfeld et al. 2006) and other anti-VEGF agents such as bevacizumab (Avastin<sup>TM</sup>, Genetech Inc, San Francisco) and pegapta&shy;nib sodium (Macugen<sup>TM</sup>)&nbsp;(Gragoudas et al. 2004) have been shown to reduce neovas&shy;cularisation in age-related macular degene&shy;ration (AMD) when injected intra-vitreally.</p>\r\n<p>\r\n	Diabetic vitrectomy is complicated by poor visualisation by the surgeon as a result of haemorrhage due to active neovascularisa&shy;tion. Post-operative vitreous haemorrhage is a further complication, resulting in de&shy;creased visual acuity as a result of bleeding particularly from optic disc neovascularisa&shy;tion, which cannot be directly cauterised.</p>\r\n<p>\r\n	A number of case series utilizing intra&shy;ocular injection of bevacizumab, a drug designed for systemic anti-cancer use as an &ldquo;off-label&rdquo; adjunct to reduce retinal neovas&shy;cularisation prior to diabetic vitrectomy have been published(Yeoh et al. 2008). Bevaci&shy;zumab has been shown to induce rapid regression of retinal and iris neovasculariza&shy;tion in proliferative diabetic retinopathy (PDR)(Avery et al. 2006).</p>\r\n<p>\r\n	Therefore, the aim of our study was to in&shy;vestigate the safety and benefit of giving an intraocular injection of ranibizumab, a drug designed and licensed for intraocular injec&shy;tion for AMD, prior to diabetic vitrectomy to reduce the intraoperative and post-operative bleeding in the eye. There has been no such study to date.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS</strong></p>\r\n<p>\r\n	A retrospective study was performed at the Universiti Kebangsaan Malaysia Medical Centre (UKMMC). The medical records of 16 diabetic patients were re&shy;viewed. These eyes received 20 consec&shy;utive ranibizumab injections from July 2007 to February 2009 prior to vitrec&shy;tomy.</p>\r\n<p>\r\n	All patients were known diabetics diag&shy;nosed by a physician and were on treat&shy;ment with including oral hypoglycaemics and/or insulin injections. Each study eye had known or suspected diabetic retino&shy;pathy. Pre-operative intravitreal ranibi&shy;zumab was considered for all patients who had never received any prior laser photocoagulation or in whom laser pho&shy;tocoagulation could not be completed due to significant areas of retinal de&shy;tachment. It was also considered for pa&shy;tients with clinically significant macular oedema secondary to vitreo-macular traction. This included patients with vi&shy;treous haemorrhage, tractional retinal detachment (TRD) with or without rheg&shy;matogenous component, vitreomacular traction (VMT) and epiretinal membrane (ERM) secondary to known or suspected proliferative diabetic retinopathy (PDR) with active neovascularisation. When the fundus could not be viewed, B scan ul&shy;trasonography was used to confirm the diagnosis.</p>\r\n<p>\r\n	Patients with prior history of cerebro&shy;vascular accident (CVA) or known vas&shy;cular occlusion of any kind were ex&shy;cluded from injection with of ranibizumab. In&shy;jection was only scheduled after the rou&shy;tine pre-operative investigations which included blood pressure reading, full blood count, renal profile, fasting blood glucose level, echocardiogram and chest X ray were acceptable for general or lo&shy;cal anaesthesia as the injection was con&shy;sidered a commitment towards operation.</p>\r\n<p>\r\n	Informed consent was obtained from each patient. This included counseling on the risk of infection, cataract and retinal detachment. Each injection was per&shy;formed in a clean treatment room in the eye clinic outpatient setting according to strict aseptic technique. Anaesthesia for the procedure was topical with guttae proparacaine hydrochloride (Alcaine<sup>TM</sup>) and subconjunctival anaesthesia with 2%<br clear="all" />\r\n	lignocaine (Xylocaine<sup>TM</sup>). Subsequently, 5% povidone iodine lid scrub and douche was performed prior to sterile draping. All patients received ranibizumab, which was aspirated by the assistant into a new 30G insulin needle after swabbing each bottle cap with 100% alcohol wipe.</p>\r\n<p>\r\n	Each injection of 0.05 or 0.1 ml of rani&shy;bizumab was given 4 mm from the lim&shy;bus in phakic patients and 3.5 mm in pseudophakic patients while directing the injection into the centre of the vitreous cavity. Five percent iodine was then re-instilled onto the ocular surface. The pa&shy;tient then received guttae ciprofloxacin eyedrops at 2 hourly intervals for one week. They were given instructions on symptoms and signs of endophthalmitis and were reviewed the following day and on the day of admission for surgery.</p>\r\n<p>\r\n	Demographical data (age, gender and ethnicity) were recorded. Dates of intra&shy;vitreal injections, volume of drug injected, surgical indications, operation record of intraoperative bleeding and duration of surgery were noted. Three cases in which anti-VEGF had been injected at any time in the past were excluded from further analysis. The pre-operative visual acuity prior to injection and on admission was noted and the post-operative visual acuity was also noted at one week, one month, 3 months, 6 months after the op&shy;eration and at last review. The clarity of ocular media was noted from the dia&shy;grams and comments in the operative notes and this was correlated with the visual acuity.</p>\r\n<p>\r\n	Post-operative haemorrhage was graded as &ldquo;nil&rdquo;, if all retina details were visible; &ldquo;mild&rdquo; if any part of the fundus was slightly obscured with blood vessels, optic disc and /or laser marks visible; &ldquo;moderate&rdquo; if any part of the fundus was totally obscured and &ldquo;severe&rdquo;, if the en&shy;tire fundus was totally obscured. The fundus under gas was used for assess&shy;ment in gas-injected eyes. Any complica&shy;tion of the intravitreal injection was noted.</p>\r\n<p>\r\n	<strong>RESULTS</strong></p>\r\n<p>\r\n	Fourteen eyes of fourteen patients re&shy;ceived intravitreal injection of 0.05 (0.5 mg) to 0.1 ml (1mg) of ranibizumab be&shy;tween 3 to 8 days prior to vitrectomy by two surgeons (Table 1). Mean duration of injection prior to surgery was 4.4 days. One patient received 1 mg of ranibizu&shy;mab. One patient received injection prior to PPV in both eyes. Patient ages ranged from 46 to 72 yearswith a mean of 57.7 years. There were 5 males and 11 fe&shy;males. Twelve patients were Malay, 3 were Chinese and 1 was Indian.Twelve were right eyes (Table 1).</p>\r\n<p>\r\n	There were no complications locally or systemically post-injection. Indications for vitrectomy were retinal detachment in 11 eyes of whom 3 were combined TRD and rhegmatogenous RD (RRD) with macula detached and 8 were tractional detach&shy;ments involving the macula. Three trac&shy;tional detachments were in combination with vitreous haemorrhage while there were eleven pre-operative vitreous hae&shy;morrhages. One eye had vitreo-macular traction syndrome secondary to PDR. One eye had re-bled 3 years after pre&shy;vious vitrectomy for TRD. Pre-operative visual acuity (VA) ranged from 6/36 to perception of light with the majority (19 eyes) with pre-operative vision of 6/60 or worse.</p>\r\n<p>\r\n	Pars planar vitrectomy (PPV) was per&shy;formed in all cases under local (n=17) or general anaesthesia (n=3) by 2 surge&shy;ons. 20G vitrectomy was performed pri&shy;marily in 15 cases and 23G in 5 cases depending on instrument availability and surgeon preference. Panretinal laser photocoagulation (PRP) was topped up or given to the ora in all cases. Intraocu&shy;lar tamponade of silicone oil 1300Cs was used in 5 cases and intraocular gas tam&shy;ponade was used in 7 cases. Of those under gas tamponade, sulphur hexafluo&shy;ride (SF6) was used in 5 cases, and oc&shy;tafluoropropane (C3F8) in 2 cases (Table 2).</p>\r\n<p>\r\n	All eyes had minimal to moderate intra&shy;operative bleeding. The amount of intra&shy;operative bleeding did not correlate with duration of injection prior to vitrectomy but more with the age of the patient and activity of neovascularisation. There was no documented case of progression of tractional detachment to RRD during the study period although fibrosis of neovas&shy;cularisation was consistently observed to various degrees in patients with view of the retina. Mild progression of the TRD not resulting in RRD accompanied fibro&shy;sis in 3 observable cases (Table 3).</p>\r\n<p>\r\n	Post-operative VH in eyes without tam&shy;ponade or with subtotal gas tamponade ranged from nil (n=1), mild (n=12) to moderate (n=1) with complete clearance 4 to 90 days post-operatively (mean 21 days). There was one case of slightly increased post-operative VH at 1 week, which cleared by 1 month during the study period.</p>\r\n<p>\r\n	Silicone oil filled eyes had nil (n=1) to moderate localized preretinal haemorr&shy;hages behind the oil (n=3) to severe post-operative haemorrhage&nbsp; (n=1), which cleared after an average of 44.8 days including one which took over 3 months to clear with oil pushed into the anterior chamber. We believe the pa&shy;tient&rsquo;s uncontrolled hypertension was re&shy;sponsible for this outcome as haemosta&shy;sis was achieved at the end of the oper&shy;ation. Two silicone filled eyes had very minimal haemorrhages and clear fundal view. Duration of follow-up ranged from 1 month (n=19) to 14 months with 18 eyes (90%) having 3 or more months of follow-up. There were 2 patients who defaulted follow-up after 1 and 3 months. (Table 3)</p>\r\n<p>\r\n	In general, post operative VH cleared in mean time of 17.6 days for 23G vitrec&shy;tomy and 30.1 days (n=10) for 20G vi&shy;trectomy (Table 3). VA had either re&shy;mained unchanged (n=2) (10%), improved (n=14) (70%) or worsened (n=4) with best corrected VA at last review ranging from 2/60 or better (n=15) to no perception of light (n=1). Two eyes achieved 6/12 or better vision (10%). Two eyes gained significant vision from hand motions or counting fingers to as much as 6/9 vision. Ten eyes (50%) achieved 6/36 or better vision. Mean gain in visual acuity was 1.8 Snellen lines. However, 60% of 23G eyes gained at least 3 Snellen lines compared to 46% of 20G eyes.</p>\r\n<p>\r\n	Two eyes (10%) developed retinal de&shy;tachment post-operatively. One patient developed total retinal detachment one-month post PPV with gas injection. He underwent further operation and retinal reattachment surgery. Another case de&shy;veloped retinal detachment following cat&shy;aract extraction and silicone oil removal. There was no emergence or exacerba&shy;tion of rubeosis in the any of the eyes at 6 months postoperatively.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Diabetic retinopathy (DR) is the leading cause of blindness among people of working age worldwide, leading to signifi&shy;cant loss of quality of life. During the first two decades of disease onset, nearly all Type 1 diabetic patients and over 60% of type 2 diabetics have retinopathy. In the Wisconsin Epidemiologic Study of Dia&shy;betic Retinopathy (WESDR), 3.6% of younger-onset patients (type 1 diabetes) and 1.6% of older-onset patients (type 2</p>\r\n', 1),
(455, 452, '<div>\r\n	<p>\r\n		<strong>INTRODUCTION </strong></p>\r\n	<p>\r\n		Only 2-4% of cholesteatomaspresentingto paediatric otologist are congenital in origin. Disease severity can range from intratympanic pearls to middle ear and mastoid obliteration (Richter &amp; Lee 2009). Derlacki &amp; Clemis (1965)in their article established the clinical criteria for the diagnosis of congenital cholestea&shy;toma (CC). These include a pearly white mass medial to an intact tympanic mem&shy;brane, a normal pars tensa and flaccida, and no previous history of otorrhoea or perforation, or previous otologic proce&shy;dure. Levenson et al. (1986) revised the criteria by adding that previous bouts of otitis media or effusion should not be the exclusion criteria. However, disease se&shy;verity depends on location, patients&rsquo; age, ossicular integrity, and number of ana&shy;tomic sites involved. These variables have inspired the development of staging systems whereby appropriate surgical approaches can be designed. Computed tomography is necessary and continue to be the best radiographic tool for surgical planning. Over 30% of CC can be extir&shy;pated through a transcanal approach. Involvement of the posterior quadrant, over three anatomic subsites, or the mastoid cavity obligates standard canal wall up techniques. Canal wall down pro&shy;cedures are rarely required (Richter &amp; Lee 2009). As demonstrated in our case, the clinical presentation of CC may not be typical as that suggested by the crite&shy;ria mentioned.</p>\r\n</div>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 6-year-old boy presented to our clinic with left ear discomfort for one year with mild reduction hearing of the ipsilateral ear. He also had recurrent episodes of left sided otorrhoea two months prior to presentation. There was copious foul smelling blood stained discharge which partially resolved with ear drops. There was no vertigo, tinnitus or facial asym&shy;metry. An otoscopic examination of the left ear revealed an intact, dull tympanic membrane and a whitish mass in the middle ear. There was posterior ear canal wall sagging with keratin debris. The tuning fork test revealed a conduc&shy;tive hearing loss on the left. Pure tone audiogram showed moderate conductive hearing loss with an air bone gap of 40dB on the left and normal hearing on the right. Tympanometry type B was found on the left and normal on the right.&nbsp; High resolution computed tomography (HRCT) of the temporal bone was performed and showed evidence of extensive choles&shy;teatoma with erosion of the scutum and ossicles (Figure 1).</p>\r\n<p>\r\n	He was subjected to elective mastoid exploration and intraoperatively, there was a large attic perforation seen with overlying keratin debris. The pars tensa was intact while the postero-superior wall of the external auditory canal was eroded. The mastoid, mesotympanum and epitympanum were filled with granu&shy;lation tissue. The ossicles including the stapes footplate were eroded and the body of the incus was enveloped by gra&shy;nulation tissue. The tegmen tympani, round window and oval window were in&shy;tact. There was dehiscence of the facial canal along the tympanic segment. The vertical, tympanic and labyrinthine seg&shy;ments of the facial canal were intact and preserved. In view of the extensiveness of the disease, he underwent radical mastoidectomy and tympanoplasty type V. Postoperatively, the patient was con&shy;tinued on intravenous rocephin and mastoid bandage for 2 days. Ribbon gauze soaked with BIPP packing was inserted and kept for 2 weeks.</p>\r\n<p>\r\n	At the time of writing, the patient remains well and has no more ear discharge. The left mastoid cavity was well epithelized and the neo-tympanic membrane was intact.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	It is well accepted that CCbehave in a more aggressive manner than the ac&shy;quired form. The most common present&shy;ing complaint is hearing reduction (Dar&shy;rouzet et al. 2002; Nelson et al 2002). As the disease progress a patient may present with vertigo, facial nerve palsy or intracranial abscess (Darrouzet et al. 2002). Early diagnosis decreases the size of CC and subsequently decreases the incidence of complications. Early de&shy;tection is possible via routine audiological or otological screening.</p>\r\n<p>\r\n	There are several theories on the pa&shy;thophysiology of congenital cholestea&shy;toma of the middle ear include presence of congenital cell rest, metaplasia of mid&shy;dle ear epithelium, and papillary ingrowth through an intact tympanic membrane (Bennett et al. 2006).&nbsp; Research reports depict that the most common location are anterior superior quadrant followed by posterior superior quadrant of the tym&shy;panic membrane (Darrouzet et al. 2002; Koltai et al. 2002; Bennet et al. 2006; Nelson et al. 2002). It is believed that these cholesteatomas behave in a more aggressive manner than the acquired form. This is based on frequent findings of more extensive invasion in a normally pneumatized temporal bone with ossicu&shy;lar and labyrinthine involvement at initial surgery (Darrouzet et al. 2002).</p>\r\n<p>\r\n	Koltai et al. (2002) reported that most cholesteatoma start out as a matrix en&shy;closed spherical keratin pearl in the ante&shy;rior superior quadrant. Accordingly, the pearl then grows like a round inflated balloon. Anterior growth is towards the eustachian tube, inferiorly towards the hypotympanum and posteriorly towards the handle of the malleus. Posterior growth may extend further, involving the incudostapedial joint and the stapes su&shy;prastructure, as well as up towards the incudomalleolar joint, around the incus, and into the attic. The growth of CCs may progress from the attic into the antrum and then into the mastoid and follows the pattern of the enlargement of other cate&shy;gories of middle ear cholesteatomas. As there have been no case reports of CCs involving the ottic capsule, Koltai et al. (2002) concluded that a young child with otorrhoea, conductive hearing loss and tympanic membrane perforation at a nontraditional location with cholestea&shy;toma filling the mastoid cavity, like in this patient, may represent the end point in the natural history of CC. This observa&shy;tion supports the notion that this case should be classified as CC. In addition, the staging system proposed by Potsic et al. (2002)(Table 1) and Nelson et al. (2002)(Table 2) would categorise this case as the last stage, namely stage 4 and type 3 respectively. In the staging system proposed by Potsic et al. (2002) extension. On the other hand, type 3 in Nelson&rsquo;s classification refers to CCs in mesotympanum with mastoid extension as well.</p>\r\n<p>\r\n	The age of this patient at presentation favours CC even though the lesion was outside the accepted definition. Findings from large series revealed that the occur&shy;rence of CC is among younger children (mean age 5.6 years) (McGill et al. 1991; Rizer &amp; Luxford 1998) compared to ac&shy;quired cholesteatoma (mean 9.7 years) (Koltai et al. 2002). The presence of tym&shy;panic membrane perforation in this pa&shy;tient does not exclude the diagnosis of CC as this may still occur with sufficient growth of CC. This is supported by the cases reported by Koltai et al. (2002) who found that CC occurs in young child&shy;ren (ages 3-6 years) presenting with sudden unrelenting otorrhoea, with little or no history of otitis media.</p>\r\n<p>\r\n	High resolution computed tomography (HRCT) of the temporal bone of these patients generally shows complete occu&shy;pation of the mastoid by soft tissue that causes osteolysis of the mastoid septa. At exploration, tympanic membrane per&shy;foration has been observed at various points in the drum other than the pars flaccida or the posterior quadrant. Accor&shy;dingly, during mastoidectomy, the entire mastoid, from antrum to tip, is host to the cholesteatoma. The presence of tym&shy;panic membrane perforation in an exten&shy;sive CC suggests 2 possible mechan&shy;isms. One is that the volume of choles&shy;teatoma exceeds the capacity of the middle ear and mastoid, resulting in tym&shy;panic membrane perforation. The other is that tympanic membrane perforation and subsequent infection accelerate the growth of the CC, resulting in complete filling of the middle ear and mastoid. Ac&shy;quired cholesteatoma can also present with otorrhoea and complete cholestea&shy;toma invasion of the middle ear and mastoid, but tympanic membrane perfo&shy;ration and otorrhoea typically occur well before the entire mastoid is involved (Koltai et al. 2002). In this case report, even though the perforation is at the pars flaccida in the posterior quadrant, it is still possible that the cholesteatoma is con&shy;genital in origin. There was no history of a chronic discharging ear or ear surgery in this patient to support that it is ac&shy;quired.</p>\r\n<p>\r\n	As mentioned earlier, recently, two staging systems for CC of the middle ear have been suggested. There was a sta&shy;tistically significant association between the stage, hearing outcomes and residual disease. In both staging systems, the more advanced the disease the higher the recurrence rate. This patient falls into stage 4 in Potsic&rsquo;s and type 3 in Nelson&rsquo;s classification. Therefore, this patient will require long term follow up. For the same reason radical mastoidectomy was per&shy;formed in this patient in order to eradi&shy;cate the cholesteatoma.</p>\r\n<p>\r\n	It had been almost two years from the operation and there were still no signs of recurrence seen at the time this manu&shy;script was written.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Early detection of CCs is crucial as it may limit the size of retrotympanic cholestea&shy;toma and reduce the possible risks and complications from surgery as well as from the disease itself.A high index of suspicion in a young child with reduced hearing and an intact tympanic mem&shy;brane may help in the diagnosis of CCs.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(456, 453, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	Dysmorphism associated with develop&shy;mental delay or mental retardation is a common indication for chromosomal analysis. Although the underlying aetiol&shy;ogy may only be recognized in ~40 to 60% of cases, there is a need to obtain an accurate diagnosis as this will affect overall management in terms of coun&shy;seling for recurrence and prognosis (Manning &amp; Hudgins 2007). Recent studies using array comparative genomic hybri&shy;dization (aCGH) have shown that dele&shy;tions and duplications are causative in 8 to 17% of such cases (Manning &amp; Hudgins 2007). We report an infant with craniofa&shy;cial dysmorphism and global develop&shy;mental delay who was diagnosed to have a terminal chromosomal deletion follow&shy;ing aCGH.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	A 3 month-old infant was referred for fa&shy;cial dysmorphism. The antenatal period was uneventful and she was born term via normal delivery. She was small for gestational age with birth weight 2.17kg. Her parents were non-consanguinous. She was admitted for neonatal jaundice at day 6 of life; newborn screening for hearing via otoacoustic emission (OAE) was abnormal. Follow-up with brainstem evoked response (BSER) revealed bila&shy;teral sensorineural hearing loss. Clini&shy;cally she was microcephalic and had fail&shy;ure to thrive. She had fair skin pigmenta&shy;tion. The ears were simple; there was hypertelorism and she had a high nasal bridge. Her nose was short; the lips were thin and the corners of her mouth were downturned (Figure 1). Her fingers were clenched and she had bila&shy;teral clinodac&shy;tyly. There was also feeding difficulty i.e. she had swallowing incoor&shy;dination and there was global develop&shy;mental delay. MRI brainshowed micro&shy;cephaly with evidence of white matter volume loss and cerebral atrophy sug&shy;gestive of perinatal ischaemic insult. Screening for inborn errors of metabol&shy;ism was negative and the preliminary conventional karyotyping (of 4 meta&shy;phase spreads) was reported as normal. In view of the craniofacial dysmorphism and global developmental delay, blood was sent for aCGH. The in&shy;vestigation revealed a 30.6Mb deletion at 5p15.33-13.3 (Figure 2) which was con&shy;firmed by fluorescence-in-situ hybridiza&shy;tion (FISH) using the Vysis LSI EGR1/D5S23, D5S721 Dual Color Probe as shown in Figure 3.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Chromosomal analysis is often re&shy;quested as a first-line investigation in a child with mental retardation, multiple congenital malformation and dysmor&shy;phism. Based on data from published research, mental retardation affects ap&shy;proximately 3% of the general popula&shy;tion; of these, less than 50% have an identifiable cause of which 40% is attri&shy;butable to chromo&shy;somal anomaly (Manning &amp; Hudgins 2007).&nbsp; In a review by Sharkey et al. 2005, 0.8% of liveborn in&shy;fants have chromosomal ano&shy;malies of whom half are associated with an ab&shy;normal phenotype (Sharkey et al. 2005). With conventional moderate level karyo&shy;typing (at 400 to 500 banding), the detection (rate of chromosomal anomalies is 13.3%; 3.8% was for structural and 7.8% for nu&shy;merical anomalies (Sharkey et al. 2005).</p>\r\n<p>\r\n	In general, both conventional and spectral karyotyping (SKY) is unreliable in detecting copy number changes of less than 5Mb (Bar-Shira et al. 2006). Array CGH is a high resolution, whole genome based technology that has a better diagnostic detection rate of these submicroscopic chromosomal aberra&shy;tions underlying patients with multiple anomaly syndromes; a resolution of &le;1Mb and up to 35kb has been reported (Manning &amp; Hudgins 2007 &amp; Bar-Shira et al. 2006). With aCGH, metaphase spreads are replaced by cloned (bacterial artificial chromosomes, BACs) or synthesized (oligonucleotides) DNA fragments across the genome that are immobilized on a glass surface, the exact chromosomal locus of which is known (Manning &amp; Hudgins 2007). Both sample and control DNA are then hybridized onto the DNA fragments spotted onto the array; copy number variations are then measured via com&shy;puter analysis based on the diferrences in hybridization pattern intensities.</p>\r\n<p>\r\n	With aCGH, the detection rate of un&shy;balanced chromosomal rearrangements has been shown to be between 10% to 24% (Bar-Shira et al. 2006). At present, there are two types of array platform: constitutional or targeted array, and whole genome array (Manning &amp; Hudgins 2007 &amp; Baldwin et al. 2008). The tar&shy;geted approach incorporates common microdeletion or duplication syndromes, telomeric regions and centromeres and selected Mendelian disorders (Baldwin et al. 2008). The development of such a custom made array has been likened to performing a &lsquo;6000 band karyotype&rsquo;. Whereas, a whole genome array has a wider coverage over the human genome. When first introduced, the whole genome array contained ~2400 BAC clones dis&shy;tributed over the genome; the newer generation of arrays are now even more dense. The resolution of aCGH is limited by the size and distance between the DNA fragments that are spotted onto the array (Manning &amp; Hudgins 2007). In a series by Baldwin et al. 2008, the genome-wide array coverage identified an additional 4.7% of clinically significant abnormalities that would not be detected by current targeted array platform. The average size of imbalances was ~3.7Mb and con&shy;tained an average of 17 known genes (Baldwin et al. 2008).&nbsp; In this study, ar&shy;ray-based CGH analysis was performed using commercially available oligonuc&shy;leotide microarrays containing about 244, 000 60-mer probes with an average res&shy;olution of about 8.9 kb (Human Genome CGH Microarray 244A kit, Agilent Tech&shy;nologies). The deletion was subsequently revealed by the use of array-based CGH, a molecular cytogenetic technique with an extremely high resolution. All these data confirm the importance of perform&shy;ing a more sophisticated investigation in patients in whom a complex phenotype is strongly suggestive of the presence of a chromosomal aberration; indeed, aCGH is a powerful technique for identifying chromosomal rearrangements where conventional cytogenetics has failed.</p>\r\n<p>\r\n	Reports of aCGH in identifying cytoge&shy;netic abnormalities missed by routine cytogenetic analyses have been reported in the literature and further support the sensitivity of aCGH (Shafer et al. 2006).In our patient, initial karyotyping was re&shy;ported as normal; interpretation was however made based on a limited num&shy;ber of metaphase spreads, as correctly cited by the laboratory. The issue of ob&shy;taining an adequate number of meta&shy;phase spreads occurs every now and then; this may be the result of insufficient volume of blood extracted which is not uncommon amongst paediatric patients particularly. Application of other molecu&shy;lar technique such as Multiplex Ligation Probe-dependant Amplification (MLPA) method can improve resolution for detec&shy;tion of changes in DNA copy number but it is typically applied in a targeted manner that assesses one or several candidate loci at a time which limit the detection rate in syndromic cases. Thus, aCGH was subsequently requested following the &lsquo;normal&rsquo; karyotype as the patient was seen to be very &lsquo;chromosomal&rsquo;, a state&shy;ment often concluded by clinical gene&shy;ticists. The latter investigation indeed eluded the underlying chromosomal anomaly which was subsequently con&shy;firmed by FISH analysis. FISH analysis is the recommended method for validating aCGH studies; furthermore, FISH is not only cost-effective but also has the ad&shy;vantage of demonstrating the mechanism of the imbalance (Baldwin et al. 2008).</p>\r\n<p>\r\n	Cri-du-chat syndrome is a relatively rare syndrome and affects between 1 in 40 000 to 50 000 live births (Cornish et al. 1999). The basic defect is due to a partial deletion, either terminal or intersti&shy;tial, of 5p15.2-p15.3. Although the name of the syndrome is derived from the high-pitched, shrill cry heard in a larger series of patients, the cry is neither pathogno&shy;monic nor present in all patients. This anomaly is largely de novo (~85%) but may be the result of an unbalanced translocation inherited from either parent (Gorlin et al. 2001). In our patient, a 30.6Mb deletion was detected at 5p15.33-p13.3; of the 54 genes that spanned this region, 10 have been re&shy;ported in association with clinical disord&shy;ers. However, two of the latter have been identified to be the most significant; this was the CTNDD2 gene and SDHA gene which are associated with Cri-du-chat syndrome and Leigh syndrome. Pheno&shy;typically, our patient showed features consistent with the former syndrome. Pa&shy;rental karyotyping and aCGH were nor&shy;mal, thus the deletion was de novo. Pre&shy;natal diagnosis with FISH using uncul&shy;tured amniocytes have been successfully reported in cases where the chromo&shy;somal anomaly is the result of a parent being a balanced translocation carrier (Gorlin et al. 2001 &amp; McKusick-Nathans Institute of Genetic Medicine 2010).</p>\r\n<p>\r\n	As anticipated, the larger the deletion, the more severe the phenotype. In addi&shy;tion to both somatic and mental retarda&shy;tion, patients with 5p deletion exhibit the following clinical features:&nbsp; microcephaly, increased inner canthal distance (75%), down-slanting palpebral fissures (60%), epicanthic folds, broad nasal bridge with prominent nasal root and a round face. Marked hypotonia and feeding difficulties are common as seen in our patient. Other major malformations include: con&shy;genital heart defects (30-50%), muscu&shy;loskeletal and central nervous system anomalies [eg hypoplastic cerebellum, corpus callosum dysgenesis and arach&shy;noid cyst] (Gorlin et al. 2001 &amp; McKusick-Nathans Institute of Genetic Medicine 2010). The MRI brain of our pa&shy;tient showed no specific structural ano&shy;maly; she is presently on a multidis&shy;cipli&shy;nary follow-up which involves regular monitoring by the developmental Paedia&shy;trician and Clinical Geneticist, rehabilita&shy;tion by occupational therapy, physiothe&shy;rapy and speech therapy, in addition to serial evaluation by audiology and oph&shy;thalmology.</p>\r\n<p>\r\n	Although aCGH is increasingly used as a diagnostic tool in patients with global developmental delay, one of the main drawbacks is its potential for identifying novel copy number variations that may not be responsible for the patient&rsquo;s global delay. Even if a variant is present in the affected individual but absent from &ldquo;nor&shy;mal&rdquo; parental genomes, it does not nec&shy;essarily follow that it is a pathogenetic change, and it may instead represent an innocuous copy-number polymorphism (a normal variation in the human genome). Interpretation of aCGH is often a chal&shy;lenging process; to facilitate this process, several international databases have been established; these include DECI&shy;PHER (Database of Chromosomal Im&shy;balance and Phenotype in Humans Us&shy;ing Ensemble Resources, http://www.sanger.ac.uk/PostGenomics/decipher/) and ECARUCA, the European Cytogeneticists Association Register of Unbalanced Chromosome Aberrations (http://www.ecaruca.net/). In a meta-anal&shy;ysis by Subramonia-Iyer et al. (2007) involving seven studies amongst patients with learning disabilities, an acceptable false-positive rate (for non-causal ab&shy;normalities) of between 5 to 10% was reported (Subramonia-Iyer et al. 2007).</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	Array CGH is a sensitive tool in detecting submicroscopic chromosomal aberra&shy;tions that are undetectable by current cytogenetic tests. However, before being accepted as a routine diagnostic evalua&shy;tion, certain issues pertaining to both clinical and laboratory aspects should be considered. These include the selection of patient phenotype, optimal array res&shy;olution and appropriateness of array platform, establishment of quality assur&shy;ance in a clinical set-up, availability of genetic counseling and of course, cost-effectiveness as compared to existing genetic tests.</p>\r\n', 1),
(457, 454, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Primary thyroid lymphoma is a rare dis&shy;order accounting for about 2% of all ma&shy;lignant lymphomas in general and less than 5% of thyroid malignancies (Sippel et al. 2002). Approximately 50-80% of pri&shy;mary thyroid lymphomas is of B-cell origin; which is the diffuse large cell type. The mucosa-associated lymphoid tissue (MALT) lymphomas, the more indolent lymphomas, account for 6 - 27% of thy&shy;roid lymphomas (Widder &amp; Pasieka 2004). The most common clinical presentation of thyroid lymphoma is a rapidly enlarging thyroid mass associated with compres&shy;sive symptoms (Sippel et al. 2002; Abdel et al. 2001) namely dyspnea, dysphagia and pain. Patients are usually elderly with a peak incidence in the sixth decade (Ha et al. 2001)and have a female prepon&shy;der&shy;ance (Sippel et al. 2002; Tupchong et al. 1986; Charles et al. 2002). These tu&shy;mours are noted to be almost always arising in the background of Hashimoto&rsquo;s thyroiditis (Sippel et al. 2002).</p>\r\n<p>\r\n	Management of thyroid lymphomas has evolved over the last 20 years from hav&shy;ing the surgeons as major players for removal of obstructive symptoms to the more cur&shy;rent approach of non-invasive chemothe&shy;rapy and external beam radia&shy;tion. Never&shy;theless, surgery still plays an important role as demonstrated by sev&shy;eral studies (Sippel et al. 2002; Widder &amp; Pasieka 2004). Optimal treatment for primary thy&shy;roid lymphoma continues to be a co&shy;lourful debate amongst clinicians.</p>\r\n<p>\r\n	<strong>CASE REPORT</strong></p>\r\n<p>\r\n	<em>Patient 1</em></p>\r\n<p>\r\n	A 65-year-old Chinese man was referred for a progressively enlarging thyroid mass of 4 months&rsquo; duration, associated with hoarseness of voice and respiratory obstructive symptoms. He had no history of fever, night sweats, loss of weight or loss of appetite.</p>\r\n<p>\r\n	On physical examination, he was a well built gentleman with a large neck swell&shy;ing. His blood pressure was 130/80 mmHg and pulse rate was 90 per minute. There were multiple enlarged cervical lymph nodes but no other abnormalities were found. His full blood count and se&shy;rum biochemistry was normal except for a mild elevation of lactate dehydroge&shy;nase (LDH) levels (644 U/L).</p>\r\n<p>\r\n	Assessment by the otolaryngologist re&shy;vealed a large multinodular goiter with retrosternal extension and bilateral vocal cord involvement. Fine needle aspiration cytology of the anterior thyroid swelling showed features consistent with poorly differentiated malignant tumour with pos&shy;sibilities that include malignant lymphoma and anaplastic carcinoma.</p>\r\n<p>\r\n	Following the biopsy, he developed sudden onset of acute obstructive respi&shy;ratory symptoms which led to an emer&shy;gency debulking of his thyroid mass and tracheostomy. The histopathological ex&shy;amination of the thyroid tissue showed diffuse infiltration by large malignant cells arranged in sheets. The malignant cells exhibit pleomorphic vesicular nuclei and occasional prominent nucleoli. Immuno&shy;histochemically, the malignant cells ex&shy;pressed B cell markers (CD20 and CD79&alpha;) and were negative for creatinine kinase and T cell markers (CD3 and CD45Ro) and this confirmed the diagno&shy;sis of diffuse large B-cell lymphoma with extra thyroidal extension.</p>\r\n<p>\r\n	Staging bone marrow aspiration and trephine biopsy showedno marrow infil&shy;tration by lymphoma cells. Computed tomography (CT) scan of the neck and thorax showed a large thyroid mass with retrosternal extension, multiple cervical lymphadenopathy, narrowing of the vocal cord and thickening of the larynx due to lymphomatous infiltration of the larynx.</p>\r\n<p>\r\n	The patient was started on chemothe&shy;rapy consisting of rituximab, cyclophos&shy;phamide, doxorubicin, vincristine and prednisolone (R-CHOP) two weeks after debulking of his tumour. During the post chemotherapy period, he developed up&shy;per gastro-intestinal bleeding, left tem&shy;poro-parietal infarct, and nosocomial pneumonia with Methicillin Resistant <em>Staphylococcus aureus</em>. However, he recovered from the complications after a prolonged stay in the hospital. After com&shy;pleting his sixth R-CHOP, a gallium scan was performed which showed an in&shy;creased in uptake in the right side of the superior mediastinum and hilar region. He was given another two cycles of R-CHOP, but a repeat gallium scan showed persistent uptake with similar pattern and intensity and then radiotherapy was planned as the next choice of treatment for this patient.</p>\r\n<p>\r\n	<em>Patient 2</em></p>\r\n<p>\r\n	A 68-year-old Malay lady who initially presented with a sudden increase in thy&shy;roid gland size, associated with pain and acute upper airway obstruction. An ur&shy;gent CT scan showed a thyroid mass with retrosternal extension and left inter&shy;nal jugular vein invasion with thrombosis. There was no mediastinal lymph node enlargement. She did not present with any B symptomssuch as fever, night sweats or loss of significant body weight. An emergency mod&shy;ified radical neck dis&shy;section was per&shy;formed and the left inter&shy;nal jugular vein was ligated. The histo&shy;pathological ex&shy;amination revealed diffuse large B-cell lymphoma of the thyroid gland (Figure 1).</p>\r\n<p>\r\n	One week later, she developed left arm swelling and pain. An ultrasound Doppler showed evidence of left subclavian and axilary vein thrombosis. She was started on warfarin tablets 2 mg daily and was referred to our centre for chemotherapy. However, she only turned up at our hos&shy;pital two weeks later with dysphagia to solid food, neck swelling and left neck pain. Physical examination showed a large left neck swelling which was firm and tender. There was also a thyroidect&shy;omy scar present. Her blood pressure was 160/90 mmHg and her pulse rate was 95 beats per minute. Examination of other systems was normal. Her full blood count and serum biochemistry were nor&shy;mal except for a highly elevated lactate dehydrogenase level (&gt; 12,000 U/L) and a slightly increased serum uric acid (577 umol/L). Her thyroid function test showed hypothyroidism (free T4: &lt;5.15 pmol/L, thyroid stimulating hor&shy;mone: 69.41 &micro;IU/ml). No bone marrow assessment was done previously.&nbsp;</p>\r\n<p>\r\n	A direct laryngoscopy showed a swol&shy;len epiglottis and left vocal cord palsy. The patient was planned for a tracheos&shy;tomy prior to chemotherapy. Unfortu&shy;nately, she collapsed in the ward a day after admission, was resuscitated, and required intensive care unit nursing. A CT angiography showed pulmonary embol&shy;ism involving the right main, ascending and descending pulmonary arteries. Her stay in the intensive care unit was com&shy;plicated by bleeding following anticoagu&shy;lant therapy, a deteriorating renal func&shy;tion, septicaemia with <em>Staphylococcus aureus</em> and hypothyroidism. Her renal function deteriorated further and she continued to bleed despite continuous support with blood products. Her coagu&shy;lation profile was persistently prolonged (PT: 22.4 seconds, INR: 1.85, APTT: &gt;180 seconds). CT scan of the brain showed gross oedema secondary to hy&shy;poxic brain injury. She finally succumbed to her illness one week post admission.</p>\r\n<p>\r\n	<strong>DISCUSSION</strong></p>\r\n<p>\r\n	Malignant tumours of the thyroid in gen&shy;eral are not very common. The incidence accounts for 1-2 cases per 100,000 people (Sippel et al. 2002). Of these, thyroid lymphomas only account for less than 5% of thyroid malignancies and 2% of all malignant lymphomas (Sippel et al. 2002). They commonly present with a rapidly enlarging neck mass, often caus&shy;ing obstructive symptoms as in both of the cases presented. It occurs more fre&shy;quently in women than men (Sippel et al. 2002; Tupchong et al. 1986; Charles et al. 2002),in keeping with the predilection of thyroid disease for women in general.</p>\r\n<p>\r\n	Thyroid lymphoma is frequently asso&shy;ciated with Hashimoto&rsquo;s thyroiditis (Abdel et al. 2001) which is not evident in both cases mentioned. There are however, case series which report the association of thyroiditis to be between 25-75% (Abdel et al. 2001). It is hypothesized that chronic antigenic stimulation secondary to autoimmune disorders leads to chronic proliferation of lymphoid cells, which eventually undergo mutation resulting in clonal proliferation and development of lymphoma (Moshynska &amp; Saxena 2008). However, not all patients with Hashi&shy;moto&rsquo;s thyroiditis develop lymphoma of the thyroid gland.</p>\r\n<p>\r\n	The International Prognostic Index (IPI) is used to predict the risk of dis&shy;ease re&shy;currence and overall survival by taking into account factors such as age, stage of disease, performance status, number of extranodal sites, and the presence or ab&shy;sence of an elevated se&shy;rum lactate dehy&shy;drogenase (LDH) (The International Non-Hodgkin&#39;s Lymphoma Prognostic Factors Project 1993). This is used gen&shy;erally to predict survival in patients with non-Hodg&shy;kin&rsquo;s lymphoma. Based on this system, both patients de&shy;scribed above have an IPI score of 2 (age and elevated LDH) which places them in the low/intermediate risk. A study done by Ha et al. (2001) tested the usefulness of the IPI in predicting the prognosis of patients with thyroid lym&shy;phoma. The 5-year sur&shy;vival rate for pa&shy;tients with an IPI of 0 was 86% versus 50% for patients with an IPI of greater than 0. The data included pa&shy;tients treated with radiation alone, che&shy;mothe&shy;rapy alone, or combined-modality ther&shy;apy. A different study (Abdel et al. 2001) of 60 patients with thyroid lym&shy;phoma showed that me&shy;diastinal involve&shy;ment and performance status was the most important indepen&shy;dent prognostic fac&shy;tors influencing re&shy;lapse-free survival and overall survival.</p>\r\n<p>\r\n	Primary thyroid lymphoma is uncom&shy;mon and its optimum management has always been a controversial issue. As the number of cases is small, mainly in the form of case reports, there is lack of ran&shy;domized prospective studies and thus different treatment protocols are being practised. In the past, surgery played a major role in the diagnosis and treatment of thyroid lymphoma (Abdel et al. 2001). This still hold true for cases of localized mucosa-associated thyroid (MALT) lym&shy;phoma where there is a complete re&shy;sponse rate of more than 90% (Widder &amp; Pasieka 2004). Surgery also plays an impor&shy;tant role in the early control of symptoms especially in patients with se&shy;vere airway obstruction or those with se&shy;vere pain (Sippel et al. 2002). A retros&shy;pective study of 46 cases by Tupchong et al. (1986) showed that patients with total macro&shy;scopic removal of the tumour had the highest rate of local control and has a long survival rate when compared to those receiving radiotherapy alone. How&shy;ever, in these cases the disease was li&shy;mited to the thyroid and cervical lymph nodes.</p>\r\n<p>\r\n	The more common type of thyroid lym&shy;phoma is diffuse large B-cell lymphoma (DLBCL) which is aggressive and usually presents with disseminated disease as shown in the present cases. The surgical risks for these patients are considerable which includes operative mortality, nerve injuries (recurrent laryngeal), hypopara&shy;thyroidism and possible damage to the trachea, esophagus and large vessels. With the advent of fine needle aspiration cytology, flow cytometry and immuno&shy;histochemical analysis; it is possible to avoid unnecessary surgical intervention for tissue diagnosis (Charles et al. 2002). Furthermore, combination therapy of radiation and chemotherapy without ex&shy;tensive surgery is associated with an equal or a superior 5 year survival rate without the additional risks of surgery (Abdel et al. 2001).</p>\r\n<p>\r\n	In the present cases, both patients un&shy;derwent emergency thyroidectomy to relieve the obstructive symptoms. In the first case, the patient received a combi&shy;nation of rituximab and CHOP. Rituximab is a chimeric monoclonal antibody that targets CD20 antigen expressed on B-cells. The use of rituximab in combination with the standard chemotherapy protocol has been shown to have significant sur&shy;vival benefit and improvement of patient outcome in DLBCL (Feugier et al. 2005). In this randomized study, rituximab com&shy;bined with CHOP was compared to CHOP alone in patients aged more than 60 years with diffuse large B-cell lym&shy;phoma and CD20-positivity. The combi&shy;nation of rituximab and CHOP improved rates of complete response, failure-free survival, and survival. Although this study does not look at primary thyroid lym&shy;phoma per se, it may be assumed that rituximab will benefit any lymphomas with CD20 positivity as it specifically targets these antigens that are present on B-cells. Our patient received 8 cycles of the combination chemotherapy and survived for 12 months from the time of presenta&shy;tion.</p>\r\n<p>\r\n	The second case was more compli&shy;cated since the patient had concomitant thrombosis, initially involving the left in&shy;ternal jugular vein which developed into a left subclavian and axillary thrombosis post-operatively. Following that, she de&shy;veloped pulmonary embolism in the ward. The acute coronary syndrome that she had prior to collapse may have been due to another thrombosis within the co&shy;ronary vessels itself. This added to the difficulty in her management. Anticoagu&shy;lant was needed to prevent further thromboembolism but she developed bleeding complications. If chemotherapy was started earlier, she might have be&shy;nefited from the reduced tumour burden. The bulk of her tumour may have contri&shy;buted to the stasis of blood within her vessels and caused the repeated epi&shy;sodes of thrombosis. A second surgery was not an option at this time as she was haemodynamically unstable. Her progno&shy;sis may have been better if chemothe&shy;rapy was started immediately after de&shy;bulking of her tumour initially.</p>\r\n<p>\r\n	&nbsp;We describe two cases of primary thy&shy;roid lymphoma that presented with an enlarging thyroid mass and had to un&shy;dergo emergency thyroidectomy. Both had similar presentations but with differ&shy;ent outcomes. Treatment options for pri&shy;mary thyroid lymphoma include com&shy;bined therapy with surgery playing a mi&shy;nimal role for diagnosis but a crucial role for compromised airway secondary to an enlarging thyroid mass. Addition of a mo&shy;noclonal antibody may increase survival risk for patients who are rapidly diag&shy;nosed and commenced early treatment.</p>\r\n', 1),
(458, 455, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Phytosterols&nbsp; or&nbsp; plant&nbsp; sterols&nbsp; are&nbsp; a&nbsp; group of&nbsp; steroid&nbsp; alcohols,&nbsp; phytochemicals naturally occuring in plants. They have a chemical&nbsp; structure&nbsp; which&nbsp; is&nbsp; similar&nbsp; to cholesterol&nbsp; (Weihrauch&nbsp; &amp;&nbsp; Gardner&nbsp; 1978) and exist in several forms in plants (Law 2000; Katan et al. 2003; Abumweis et al. 2007) including &beta;-sitosterol, campesterol, stigmasterol&nbsp; and&nbsp; cycloartenol&nbsp; (Ostlund 2002).&nbsp; Phytosterols&nbsp; are&nbsp; natural&nbsp; compo- nents&nbsp; found&nbsp; in&nbsp; the&nbsp; human&nbsp; diet.&nbsp; It&nbsp; made up&nbsp; 0.1&nbsp; to&nbsp; 0.5%&nbsp; w/w&nbsp; of&nbsp; vegetable&nbsp; oils&nbsp; or margerine&nbsp; (Kochlar&nbsp; 1983)&nbsp; and&nbsp; are&nbsp; also found in corn, wheat and rice. Phytosterol intake varies according to the type of diet taken.&nbsp; Europeans&nbsp; consume&nbsp; about&nbsp; 200- 300&nbsp; mg/day&nbsp; of&nbsp; phytosterol&nbsp; (Morton&nbsp; et&nbsp; al. 1995)&nbsp; while&nbsp; the&nbsp; vegetarian&nbsp; Japanese have&nbsp; a&nbsp; higher&nbsp; intake&nbsp; of&nbsp; phytosterol&nbsp; (Nair et al. 1984).&nbsp;</p>\r\n<p>\r\n	Phytosterols&nbsp; are&nbsp; well&nbsp; known&nbsp; for&nbsp; their ability&nbsp; to lower&nbsp; plasma&nbsp; cholesterol&nbsp; level (Klingberg et al. 2008) by interfering with the&nbsp; absorption&nbsp; of&nbsp; cholesterol&nbsp; from&nbsp; the gastrointestinal&nbsp; system&nbsp; (Jones&nbsp; 1999; Hayes&nbsp; et al.&nbsp; 2004;&nbsp; Jia&nbsp; et al.&nbsp; 2007).</p>\r\n<p>\r\n	Phytosterols&nbsp; have&nbsp; also&nbsp; been&nbsp; shown&nbsp; to possess&nbsp; anti-cancer&nbsp; properties&nbsp; against gastric,&nbsp; colon,&nbsp; ovary&nbsp; and&nbsp; breast&nbsp; cancers (Choi&nbsp; et al. 2007;&nbsp; Mendilaharsu&nbsp; et al. 1998; De Stefani et al. 2000; McCann et al.&nbsp; 2003;&nbsp; Ju&nbsp; et&nbsp; al.&nbsp; 2004).&nbsp; These&nbsp; studies suggested&nbsp; that phytosterols&nbsp; may&nbsp; be useful in the prevention of cardiovascular diseases and cancers.</p>\r\n<p>\r\n	Oxidative&nbsp; stress&nbsp; is&nbsp; thought&nbsp; to be&nbsp; in- volved&nbsp; in the&nbsp; pathogenesis&nbsp; of coronary and peripheral arterial diseases (Smith et al. 1993). During lipid peroxidation, lipids are&nbsp; oxidized&nbsp; to&nbsp; form&nbsp; free&nbsp; radicals&nbsp; which can&nbsp; cause&nbsp; extensive&nbsp; tissue&nbsp; damage. Several&nbsp; cardiovascular&nbsp; risk&nbsp; factors&nbsp; have been&nbsp; identified&nbsp; that&nbsp; promote&nbsp; cardiovas- cular&nbsp; disease&nbsp; via&nbsp; lipid&nbsp; peroxidation (Rumley et al. 2004). Therefore, reducing lipid&nbsp; peroxidation&nbsp; is&nbsp; important&nbsp; in preven- tion&nbsp; of&nbsp; cardiovascular&nbsp; disease.&nbsp; This&nbsp; may be&nbsp; achieved&nbsp; through&nbsp; the&nbsp; anti-oxidant properties&nbsp; of&nbsp; phytosterols.&nbsp; It is&nbsp; also&nbsp; be- lieved&nbsp; that&nbsp; oxidative&nbsp; stress&nbsp; may&nbsp; contri- bute&nbsp; to&nbsp; carcinogenesis&nbsp; by&nbsp; DNA.&nbsp; How- ever, there is a growing body of evidence showing&nbsp; that lipid&nbsp; peroxidation&nbsp; do not promote carcinogenesis but may actually inhibit&nbsp; most&nbsp; cancer&nbsp; cells&nbsp; (Zanetti&nbsp; et&nbsp; al. 2003; Gago-Dominguez et al. 2005).&nbsp;</p>\r\n<p>\r\n	Phytosterols&nbsp; were&nbsp; found&nbsp; to&nbsp; exert&nbsp; anti- oxidant effects on the oxidation of methyl linoleate&nbsp; in&nbsp; solution.&nbsp; They&nbsp; also&nbsp; sup- pressed&nbsp; the&nbsp; oxidation&nbsp; and&nbsp; consumption of&nbsp; &alpha;-tocopherol&nbsp; in&nbsp; &beta;-linoleoyl-&gamma;-palmitoyl phosphatidylcholine&nbsp; (PLPC)&nbsp; liposomal membranes&nbsp; (Yoshida&nbsp; &amp;&nbsp; Niki&nbsp; 2003).&nbsp; Vi- vancos and Moreno (2005) reported that phytosterols&nbsp; were&nbsp; able&nbsp; to&nbsp; increase&nbsp; the anti-oxidant&nbsp; enzyme&nbsp; activities&nbsp; of&nbsp; supe- roxide&nbsp; dismutase&nbsp; and&nbsp; glutathione&nbsp; perox- idase&nbsp; in&nbsp; cultured&nbsp; macrophage&nbsp; cells&nbsp; ex- posed&nbsp; to&nbsp; oxidative&nbsp; stress&nbsp; by&nbsp; phorbol&nbsp; 12- myristate 13-acetate. Therefore, an alter- native&nbsp; mechanism&nbsp; of&nbsp; protection&nbsp; from oxidative&nbsp; stress&nbsp; by&nbsp; phytosterols&nbsp; is&nbsp; by&nbsp; in- creasing&nbsp; the&nbsp; antioxidant&nbsp; enzyme&nbsp; activities.</p>\r\n<p>\r\n	There were limited published reports on the&nbsp; effects&nbsp; of&nbsp; phytosterol&nbsp; on&nbsp; lipid&nbsp; peroxi- dation.&nbsp; The&nbsp; aim&nbsp; of&nbsp; our&nbsp; study&nbsp; is&nbsp; to&nbsp; deter- mine&nbsp; the&nbsp; effects&nbsp; of&nbsp; pretreatment&nbsp; with phytosterol&nbsp; on&nbsp; lipid&nbsp; peroxidation&nbsp; in plasma&nbsp; and&nbsp; organs&nbsp; of&nbsp; rats&nbsp; exposed&nbsp; to carbon tetrachloride. &nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS&nbsp; </strong></p>\r\n<p>\r\n	A&nbsp; preliminary&nbsp; study&nbsp; was&nbsp; conducted&nbsp; to determine&nbsp; the&nbsp; suitable&nbsp; dose&nbsp; of&nbsp; carbon tetrachloride&nbsp; which&nbsp; can&nbsp; induce&nbsp; lipid&nbsp; pe- roxidation&nbsp; in&nbsp; various&nbsp; rat&nbsp; organs.&nbsp; Hafsah (2005) found that the dose of 1.0 ml/kg of carbon&nbsp; tetrachloride overwhelmed&nbsp; the effects&nbsp; of&nbsp; phytosterols&nbsp; which&nbsp; were&nbsp; given at&nbsp; weekly&nbsp; doses&nbsp; of&nbsp; 140&nbsp; mg/kg&nbsp; (unpublished).&nbsp;</p>\r\n<p>\r\n	In&nbsp; this&nbsp; study,&nbsp; 24 male&nbsp; Sprague-Dawley rats weighing between 175 to 200 grams were&nbsp; obtained&nbsp; from&nbsp; the&nbsp; UKM&nbsp; Animal House.&nbsp; The&nbsp; rats&nbsp; were&nbsp; housed&nbsp; in plastic cages at room temperature (29&plusmn;3&ordm;C) and daily&nbsp; dark/light&nbsp; cycle.&nbsp; The&nbsp; rats&nbsp; were&nbsp; fed standard rat pellets (Gold Coin, Malaysia) and&nbsp; distilled&nbsp; water&nbsp; ad libitum.&nbsp; The&nbsp; rats were&nbsp; allowed&nbsp; to adjust&nbsp; to the new&nbsp; envi- ronment for a week before the study was started.&nbsp; The&nbsp; study&nbsp; was&nbsp; approved&nbsp; by&nbsp; the UKM Animal Ethics Committee.</p>\r\n<p>\r\n	The&nbsp; rats&nbsp; were&nbsp; randomly&nbsp; divided&nbsp; into&nbsp; 4 groups&nbsp; of&nbsp; normal&nbsp; control&nbsp; (NC),&nbsp; carbon tetrachloride&nbsp; (CCl4),&nbsp; phytosterol&nbsp; (P)&nbsp; and phytosterol+carbon&nbsp; tetrachloride (P+CCl4).&nbsp; The&nbsp; latter&nbsp; two&nbsp; groups&nbsp; were pretreated&nbsp; with&nbsp; a subcutaneous&nbsp; injection of&nbsp; phytosterol&nbsp; (MPOB,&nbsp; Malaysia)&nbsp; at a dose of 140 mg/kg, once a week for five weeks&nbsp; (Yoshida&nbsp; &amp;&nbsp; Niki&nbsp; 2003).&nbsp; These phytosterols were extracted from palm oil and&nbsp; are&nbsp; composed&nbsp; of&nbsp; 60%&nbsp; &beta;-sitosterol and&nbsp; 40%&nbsp; stigmasterol&nbsp; and&nbsp; campesterol. The&nbsp; NC&nbsp; and&nbsp; CCl4&nbsp; groups&nbsp; received&nbsp; 2 ml/kg&nbsp; olive oil&nbsp; (vehicle)&nbsp; (Oomerbhoy&nbsp; Ltd, Mumbai)&nbsp; subcutaneously&nbsp; once&nbsp; a&nbsp; week for&nbsp; the same&nbsp; duration.&nbsp; Rats&nbsp; in the&nbsp; CCl4 and&nbsp; P + CCl4&nbsp; groups&nbsp; were&nbsp; then given&nbsp; a single dose of 0.5 ml/kg carbon tetrachlo- ride (BDH Chemicals, England) diluted in olive&nbsp; oil&nbsp; via&nbsp; oral&nbsp; gavage&nbsp; to&nbsp; induce&nbsp; lipid peroxidation.&nbsp; Rats&nbsp; in the&nbsp; NC&nbsp; and&nbsp; P groups&nbsp; received&nbsp; equivalent&nbsp; amounts&nbsp; of olive&nbsp; oil&nbsp; (vehicle).&nbsp; After&nbsp; 24&nbsp; hours,&nbsp; blood samples&nbsp; were&nbsp; taken&nbsp; via the&nbsp; orbital&nbsp; sinus under anesthesia. The blood&nbsp; was left for 30&nbsp; minutes&nbsp; and&nbsp; centrifuged&nbsp; at 3000&nbsp; rpm for&nbsp; 5 minutes&nbsp; to&nbsp; separate&nbsp; the&nbsp; plasma. The&nbsp; plasma&nbsp; was&nbsp; stored&nbsp; at -20&deg;C.&nbsp; The rats&nbsp; were&nbsp; then&nbsp; sacrificed&nbsp; and the&nbsp; liver, heart,&nbsp; kidneys&nbsp; and&nbsp; lungs&nbsp; were&nbsp; dissected out.&nbsp; The&nbsp; plasma&nbsp; and&nbsp; tissue&nbsp; ma- londialdehyde&nbsp; (MDA)&nbsp; levels&nbsp; were&nbsp; meas- ured&nbsp; according&nbsp; to&nbsp; the&nbsp; method&nbsp; of&nbsp; Led- wozyw&nbsp; et al.&nbsp; (1986).&nbsp; According&nbsp; to this method,&nbsp; malondialdehyde&nbsp; is&nbsp; formed&nbsp; as an end product of lipid peroxidation which reacts&nbsp; with&nbsp; TBA&nbsp; (Thiobarbutaric&nbsp; acid) reagent&nbsp; under&nbsp; acidic&nbsp; condition&nbsp; to gener- ate a pink-coloured product. Plasma (0.1 ml) was added to 0.4 ml of distilled water, followed&nbsp; by&nbsp; the&nbsp; addition&nbsp; of&nbsp; 2.5 ml&nbsp; of trichloroacetic&nbsp; acid&nbsp; (TCA).&nbsp; The&nbsp; mixture was&nbsp; left&nbsp; at room&nbsp; temperature&nbsp; for&nbsp; 15 mi- nutes. TBA (1.5 ml) was then added and heated&nbsp; in a water&nbsp; bath&nbsp; at&nbsp; 100&deg;C&nbsp; for&nbsp; 30 minutes&nbsp; until&nbsp; a faint&nbsp; pink&nbsp; colour&nbsp; was&nbsp; ob- tained.&nbsp; After&nbsp; cooling,&nbsp; the colour&nbsp; was&nbsp; extracted in 1 ml of buthanol and the inten- sity&nbsp; was&nbsp; measured&nbsp; using&nbsp; the&nbsp; spectro- photometer&nbsp; at&nbsp; EX&nbsp; 515nm&nbsp; and&nbsp; EM 553nm.&nbsp; 1,1,1,3-tetraethoxypropane (Sigma, USA) was used as the standard. &nbsp;&nbsp; The&nbsp; results are expressed as&nbsp; mean&plusmn;SEM. The statistical significance of the&nbsp; data&nbsp; was&nbsp; determined&nbsp; using&nbsp; one-way analysis&nbsp; of&nbsp; variance&nbsp; (ANOVA)&nbsp; and&nbsp; post hoc&nbsp; Tukey&nbsp; test.&nbsp; The&nbsp; level&nbsp; of&nbsp; significance was set at p&lt;0.05.&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS&nbsp; </strong></p>\r\n<p>\r\n	In order to determine the suitable dose of carbon&nbsp; tetrachloride&nbsp; for&nbsp; induction&nbsp; of&nbsp; lipid peroxidation,&nbsp; we&nbsp; have&nbsp; carried&nbsp; out a pre- liminary&nbsp; study.&nbsp; In the study,&nbsp; rats&nbsp; were given&nbsp; either&nbsp; 0.5 ml/kg&nbsp; or&nbsp; 0.75 ml/kg&nbsp; of carbon&nbsp; tetrachloride&nbsp; and&nbsp; sacrificed&nbsp; after 24&nbsp; hours.&nbsp; The&nbsp; plasma&nbsp; and liver&nbsp; melon- dialdehyde&nbsp; (MDA)&nbsp; were&nbsp; then&nbsp; measured. We found from our preliminary study that the&nbsp; lowest&nbsp; dose&nbsp; of&nbsp; carbon&nbsp; tetrachloride that&nbsp; would&nbsp; produce&nbsp; significant&nbsp; lipid&nbsp; pe- roxidation&nbsp; in&nbsp; plasma&nbsp; and liver&nbsp; was&nbsp; 0.5 ml/kg&nbsp; (Figures&nbsp; 1 and&nbsp; 2).&nbsp; Results&nbsp; of&nbsp; the present&nbsp; study&nbsp; showed&nbsp; that&nbsp; the&nbsp; plasma MDA of carbon tetrachloride (CCl4) group was&nbsp; significantly&nbsp; higher&nbsp; than&nbsp; the&nbsp; normal control group. The plasma MDA levels of the&nbsp; P and P+CCl4&nbsp; groups&nbsp; were&nbsp; signifi- cantly lower than the CCl4 group (Figure 3).&nbsp; The&nbsp; hepatic&nbsp; MDA&nbsp; level&nbsp; for&nbsp; the&nbsp; CCl4 group&nbsp; was&nbsp; significantly&nbsp; higher&nbsp; than&nbsp; the normal&nbsp; control&nbsp; group.&nbsp; The&nbsp; hepatic&nbsp; MDA levels of the P and P+CCl4&nbsp; groups&nbsp; were significantly&nbsp; lower&nbsp; than&nbsp; the&nbsp; CCl4&nbsp; groups. In&nbsp; addition,&nbsp; the&nbsp; hepatic&nbsp; MDA&nbsp; level&nbsp; of&nbsp; P group&nbsp; was&nbsp; significantly&nbsp; lower&nbsp; than the normal&nbsp; control&nbsp; group&nbsp; (Figure&nbsp; 4).&nbsp; There was&nbsp; no significant&nbsp; change&nbsp; in renal&nbsp; MDA level&nbsp; in&nbsp; the&nbsp; CCl4&nbsp; group&nbsp; compared&nbsp; to the normal&nbsp; control&nbsp; group.&nbsp; The&nbsp; renal&nbsp; MDA levels for the P and P+CCl4 groups were significantly&nbsp; lower&nbsp; compared&nbsp; to the CCl4 group&nbsp; (Figure&nbsp; 5).&nbsp; There&nbsp; was&nbsp; no&nbsp; signifi- cant&nbsp; change&nbsp; in the&nbsp; cardiac&nbsp; MDA&nbsp; level&nbsp; in the CCl4 group compared to normal control&nbsp; group. The&nbsp; cardiac MDA&nbsp; level for&nbsp; the P group was significantly lower than P and P+CCl4 &nbsp; groups&nbsp; (Figure&nbsp; 6).&nbsp; The&nbsp; MDA&nbsp; le- vels&nbsp; in&nbsp; the&nbsp; lungs&nbsp; showed&nbsp; no&nbsp; significant findings in all the groups (Figure 7).&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	Carbon&nbsp; tetrachloride&nbsp; is&nbsp; metabolised&nbsp; by cytochrome P-450 enzymes in the liver to the&nbsp; reactive&nbsp; trichloromethyl&nbsp; radical.&nbsp; The radical&nbsp; is&nbsp; oxidized&nbsp; further,&nbsp; forming&nbsp; the even&nbsp; more&nbsp; reactive&nbsp; trichloromethylpe- roxyl radical (McGregor &amp; Lang 1996;&nbsp;&nbsp; IPCS&nbsp; 1999).&nbsp; These&nbsp; reactive&nbsp; metabolic intermediates&nbsp; of&nbsp; carbon&nbsp; tetrachloride, particularly trichloromethylperoxyl radical, can cause lipid peroxidation (IPCS 1999). In&nbsp; our&nbsp; study,&nbsp; carbon&nbsp; tetrachloride&nbsp; has induced&nbsp; lipid&nbsp; peroxidation&nbsp; as&nbsp; shown&nbsp; by elevations&nbsp; of&nbsp; MDA&nbsp; levels&nbsp; in&nbsp; the&nbsp; rat&rsquo;s plasma,&nbsp; liver,&nbsp; kidneys,&nbsp; heart&nbsp; and&nbsp; lungs. However,&nbsp; only&nbsp; the&nbsp; plasma&nbsp; and&nbsp; hepatic MDA&nbsp; levels&nbsp; were&nbsp; significantly&nbsp; raised. to&nbsp; cause injuries&nbsp; to&nbsp; various&nbsp; organs&nbsp; of&nbsp; the&nbsp; body (Reynolds et al. 1984) including the liver, kidneys,&nbsp; heart,&nbsp; lungs,&nbsp; gastrointestinal tract&nbsp; and&nbsp; central&nbsp; nervous&nbsp; system (ATSDR&nbsp; 2005).&nbsp; The&nbsp; primary&nbsp; targets&nbsp; for carbon&nbsp; tetrachloride&nbsp; toxicity&nbsp; are&nbsp; the&nbsp; liver and kidneys (IPCS 1999).</p>\r\n<p>\r\n	Phytosterols taken into the body are in- corporated into cell membranes (Awad et al.&nbsp; 2004)&nbsp; and&nbsp; are&nbsp; highly&nbsp; concentrated&nbsp; in the&nbsp; lungs,&nbsp; adrenal&nbsp; cortex,&nbsp; intestinal&nbsp; epithelia and&nbsp; ovaries (Sanders et al.&nbsp; 2000). In our study, we find that phytosterol pre- treatment&nbsp; was&nbsp; able&nbsp; to prevent&nbsp; elevation of&nbsp; MDA&nbsp; levels&nbsp; in the&nbsp; plasma,&nbsp; liver&nbsp; and kidneys&nbsp; but&nbsp; was&nbsp; unable&nbsp; to&nbsp; do&nbsp; the&nbsp; same for&nbsp; the heart&nbsp; and&nbsp; lungs.&nbsp; Therefore,&nbsp; pretreatment&nbsp; with&nbsp; phytosterol&nbsp; was&nbsp; able&nbsp; to reduce&nbsp; lipid&nbsp; peroxidation&nbsp; in&nbsp; the&nbsp; plasma and protect the liver and kidneys against lipid&nbsp; peroxidation&nbsp; damage.&nbsp; These&nbsp; find- ings&nbsp; were&nbsp; consistent&nbsp; with&nbsp; other&nbsp; findings that showed that pretreatment&nbsp; with other antioxidants,&nbsp; such&nbsp; as&nbsp; vitamin&nbsp; E reduced the&nbsp; hepatotoxic&nbsp; action&nbsp; of&nbsp; carbon tetrach- loride (IPCS 1999).&nbsp;</p>\r\n<p>\r\n	In&nbsp; a study&nbsp; by&nbsp; Mora-Ranjeva&nbsp; et al. (2006), phytosterol in the form of sitoste- rol&nbsp; and&nbsp; stigmasterol&nbsp; were&nbsp; incorporated into&nbsp; human&nbsp; keratinocytes&nbsp; (SVK14&nbsp; line) and&nbsp; exposed&nbsp; to ultraviolet&nbsp; light.&nbsp; It was found&nbsp; that sitosterol&nbsp; induced&nbsp; significant decrease&nbsp; (-30%)&nbsp; in&nbsp; lipid&nbsp; peroxidation whereas stigmasterol markedly increased lipid peroxidation (+70%). Results of this&nbsp; study&nbsp; also&nbsp; showed&nbsp; that&nbsp; the&nbsp; effects&nbsp; of phytosterol on lipid peroxidation also de- pended on the form of the phytosterol. In this&nbsp; study,&nbsp; we have&nbsp; used&nbsp; phytosterols derived from palm oil which was made up of 60% &beta;-sitosterol and 40% campesterol and stigmasterol.</p>\r\n<p>\r\n	We&nbsp; found&nbsp; that&nbsp; there&nbsp; were&nbsp; measurable MDA&nbsp; levels in the plasma and organs of rats&nbsp; in the normal&nbsp; control&nbsp; group.&nbsp; This may&nbsp; be&nbsp; contributed&nbsp; by&nbsp; the&nbsp; free&nbsp; radicals being&nbsp; produced&nbsp; even&nbsp; under&nbsp; normal&nbsp; con- ditions&nbsp; by&nbsp; either&nbsp; leakage&nbsp; of&nbsp; activated oxygen&nbsp; from&nbsp; mitochondria&nbsp; during&nbsp; oxida- tive&nbsp; phosphorylation&nbsp; or&nbsp; by&nbsp; the&nbsp; multiple redox-active&nbsp; flavoproteins&nbsp; (Messner&nbsp; &amp; Imlay&nbsp; 2002;&nbsp; Imlay&nbsp; 2003;&nbsp; Seaver&nbsp; &amp;&nbsp; Imlay 2004).&nbsp; Phytosterol supplementation was able to reduce the MDA levels of the liver and&nbsp; heart&nbsp; of&nbsp; normal&nbsp; rats.&nbsp; Based&nbsp; on this result, phytosterol may be useful as sup- plements for&nbsp; healthy&nbsp; individuals&nbsp; to main- tain their oxidative status but this requires further studies.&nbsp;</p>\r\n<p>\r\n	The&nbsp; phytosterol&nbsp; dosage&nbsp; used&nbsp; in&nbsp; this study is equivalent to the human intake of 20&nbsp; mg/kg&nbsp; body&nbsp; weight/day.&nbsp; This&nbsp; is&nbsp; high considering&nbsp; that&nbsp; the&nbsp; daily&nbsp; intake&nbsp; of&nbsp; phy- tosterols in the human diet is about 2.5 to 4.0&nbsp; mg/kg&nbsp; bodyweight&nbsp; (Morton&nbsp; et al. 1995).&nbsp; With&nbsp; regards&nbsp; to its&nbsp; toxicity,&nbsp; ad-</p>\r\n<p>\r\n	ministration&nbsp; of&nbsp; phytosterols&nbsp; at the&nbsp; daily doses&nbsp; of&nbsp; 6.6 g/kg&nbsp; bodyweight&nbsp; in rats&nbsp; did not show any subchronic toxicity or tera- togenic effects (Hepburn et al. 1999). In a human&nbsp; study,&nbsp; the&nbsp; daily&nbsp; intake&nbsp; of&nbsp; 9.0 grams of phytosterols for eight weeks did not&nbsp; cause&nbsp; any&nbsp; adverse&nbsp; effects&nbsp; (Katan&nbsp; et al.&nbsp; 2003).&nbsp; However,&nbsp; it&nbsp; was&nbsp; shown&nbsp; to&nbsp; re- duce&nbsp; absorption&nbsp; of&nbsp; lipid&nbsp; soluble&nbsp; antioxi- dants&nbsp; such&nbsp; as&nbsp; &alpha;-carotene,&nbsp; &beta;-carotene and vitamin E (Law 2000).</p>\r\n<p>\r\n	In&nbsp; conclusion,&nbsp; phytosterols&nbsp; may&nbsp; be used&nbsp; to&nbsp; reduce&nbsp; lipid&nbsp; peroxidation&nbsp; in&nbsp; the plasma,&nbsp; liver&nbsp; and&nbsp; kidneys.&nbsp; Phytosterol supplementation&nbsp; has&nbsp; a&nbsp; good&nbsp; safety&nbsp; pro- file&nbsp; and&nbsp; may&nbsp; further&nbsp; reduce&nbsp; lipid&nbsp; peroxi- dation in healthy individuals.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS&nbsp; </strong></p>\r\n<p>\r\n	We&nbsp; would&nbsp; like&nbsp; to&nbsp; thank&nbsp; the&nbsp; staff&nbsp; of&nbsp; the Pharmacology Department for their tech- nical assistance.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(459, 456, '<p>\r\n	<strong>INTRODUCTION&nbsp; </strong></p>\r\n<p>\r\n	Breast&nbsp; cancer&nbsp; is&nbsp; the&nbsp; most&nbsp; common&nbsp; malignancy&nbsp; that&nbsp; develops&nbsp; in&nbsp; women&nbsp; worldwide,&nbsp; its&nbsp; incidence&nbsp; continues&nbsp; to&nbsp; rise&nbsp; and it&nbsp; is&nbsp; responsible&nbsp; for&nbsp; the&nbsp; highest&nbsp; death rates&nbsp; (Kamangar&nbsp; et&nbsp; al.&nbsp; 2006).&nbsp; Breast cancer&nbsp; is&nbsp; a&nbsp; disease&nbsp; with&nbsp; complex&nbsp; aetiology&nbsp; with&nbsp; multiple&nbsp; predisposing&nbsp; factors. There&nbsp; is&nbsp; a&nbsp; substantial&nbsp; body&nbsp; of&nbsp; experimental,&nbsp; clinical&nbsp; and&nbsp; epidemiological&nbsp; evidence&nbsp; indicating&nbsp; that&nbsp; hormones&nbsp; play&nbsp; a major&nbsp; role&nbsp; in&nbsp; the&nbsp; development&nbsp; of&nbsp; breast cancer and this has been extensively reviewed&nbsp; in&nbsp; the&nbsp; literature&nbsp; (Clarke&nbsp; &amp; Sutherland&nbsp; 1990;&nbsp; Clarkson&nbsp; et&nbsp; al.&nbsp; 2004; Henderson &amp; Feigelson 2000; Pike et al. 1993;&nbsp; Russo&nbsp; &amp;&nbsp; Russo&nbsp; 1998).&nbsp; Firstly, breast cancer in men is rare, suggesting an&nbsp; influence&nbsp; of&nbsp; the&nbsp; female&nbsp; sex&nbsp; steroids (Hulka&nbsp; &amp;&nbsp; Stark&nbsp; 1995).&nbsp; Secondly,&nbsp; in&nbsp; studies&nbsp; conducted&nbsp; as&nbsp; early&nbsp; as&nbsp; 1896,&nbsp; it&nbsp; was reported&nbsp; that&nbsp; removal&nbsp; of&nbsp; the&nbsp; ovaries caused&nbsp; the&nbsp; regression&nbsp; of&nbsp; breast&nbsp; cancer, and&nbsp; subsequent&nbsp; studies,&nbsp; many&nbsp; years later,&nbsp; reported&nbsp; that&nbsp; the&nbsp; estrogen&nbsp; antagonist, Tamoxifen, had proven efficacy in the treatment of advanced breast cancer and in the reduction of breast cancer risk when used in an adjuvant setting (Cuzick et&nbsp; al.&nbsp; 2003;&nbsp; Jordan&nbsp; 2003;&nbsp; Russo&nbsp; and Russo 1998). Thirdly, it has been repeatedly&nbsp; demonstrated&nbsp; that&nbsp; estrogen&nbsp; and progesterone&nbsp; treatment&nbsp; in&nbsp; animal&nbsp; models,&nbsp; including&nbsp; rodents&nbsp; and&nbsp; monkeys,&nbsp; can promote&nbsp; mammary&nbsp; carcinogenesis&nbsp; and this&nbsp; is&nbsp; critically&nbsp; dependent&nbsp; on&nbsp; functional estrogen&nbsp; and&nbsp; progesterone&nbsp; receptors (ER&nbsp; and&nbsp; PR)&nbsp; (Clarke&nbsp; and&nbsp; Sutherland 1990;&nbsp; Clarkson&nbsp; et&nbsp; al.&nbsp; 2004;&nbsp; Russo&nbsp; and Russo&nbsp; 1998).&nbsp; Finally,&nbsp; there&nbsp; have&nbsp; been numerous&nbsp; epidemiological&nbsp; studies&nbsp; which have reported increases in breast cancer risk in women with increased exposure to endogenous&nbsp; and&nbsp; exogenous&nbsp; hormones (Santen&nbsp; 2003;&nbsp; Writing&nbsp; Group&nbsp; for&nbsp; the Women&#39;s&nbsp; Health&nbsp; Initiative&nbsp; Investigators 2002).&nbsp; Endogenous&nbsp; and&nbsp; exogenous hormones can promote tumour formation by driving cell proliferation, subsequently increasing the number of cell divisions as well&nbsp; as&nbsp; the&nbsp; opportunity&nbsp; for&nbsp; random&nbsp; genetic&nbsp; errors&nbsp; (Henderson&nbsp; and Feigelson 2000).&nbsp; Alternatively,&nbsp; estrogen&nbsp; metabolites may directly generate DNA damage, thus&nbsp; leading&nbsp; to&nbsp; genomic&nbsp; instability&nbsp; (Liehr 2000; Yue et al. 2003). However, there is also&nbsp; strong&nbsp; evidence&nbsp; for&nbsp; a&nbsp; genetic&nbsp; component&nbsp; to breast&nbsp; cancer,&nbsp; the strongest risk&nbsp; factor&nbsp; today&nbsp; is&nbsp; a family&nbsp; history.&nbsp; The genetic&nbsp; basis&nbsp; for&nbsp; the inherited&nbsp; predisposition to breast cancer has been ardently investigated in the past two decades and has&nbsp; resulted&nbsp; in&nbsp; the discovery&nbsp; of&nbsp; several high- to low-penetrance&nbsp; breast&nbsp; cancer susceptibility&nbsp; genes&nbsp; through&nbsp; genome-wide&nbsp; linkage&nbsp; studies&nbsp; and&nbsp; mutational screening&nbsp; of&nbsp; candidate&nbsp; genes&nbsp; in large breast&nbsp; cancer&nbsp; case&nbsp; series.&nbsp; This&nbsp; review will&nbsp; discuss&nbsp; the known&nbsp; genetic&nbsp; risk&nbsp; fac- tors&nbsp; for&nbsp; developing&nbsp; breast&nbsp; cancer&nbsp; with&nbsp; a particular focus on BRCA1.</p>\r\n<p>\r\n	<strong>HIGHLY&nbsp; PENETRANT&nbsp; BREAST CANCER SUSCEPTIBILITY GENES&nbsp; </strong></p>\r\n<p>\r\n	<em>BRCA1/2&nbsp;&nbsp; </em></p>\r\n<p>\r\n	Hereditary&nbsp; breast&nbsp; cancer&nbsp; accounts&nbsp; for&nbsp; a small&nbsp; but&nbsp; significant&nbsp; proportion&nbsp; (12.9%) of&nbsp; all&nbsp; breast&nbsp; cancers&nbsp; (Collaborative Group&nbsp; on&nbsp; Hormonal&nbsp; Factors&nbsp; in&nbsp; Breast Cancer&nbsp; 2001).&nbsp; Analysis&nbsp; of&nbsp; pedigrees from high-risk breast/ovarian cancer fam- ilies&nbsp; led&nbsp; to&nbsp; the&nbsp; identification&nbsp; and&nbsp; cloning of&nbsp; the&nbsp; first&nbsp; breast&nbsp; cancer&nbsp; susceptibility gene&nbsp; (BRCA1)&nbsp; (Hall&nbsp; et&nbsp; al.&nbsp; 1990;&nbsp; Miki&nbsp; et al.&nbsp; 1994)&nbsp; spanning&nbsp; approximately&nbsp; 100kb of&nbsp; genomic&nbsp; DNA&nbsp; and&nbsp; consisting&nbsp; of&nbsp; 24 exons (although exons 1 and 4 are non- coding),&nbsp; that&nbsp; encode&nbsp; a&nbsp; large&nbsp; multi-domain protein of 1863aa (Miki et al. 1994). The&nbsp; role&nbsp; of&nbsp; this&nbsp; protein&nbsp; in&nbsp; genetic&nbsp; susceptibility&nbsp; to&nbsp; breast&nbsp; and&nbsp; ovarian&nbsp; cancer was&nbsp; confirmed&nbsp; when&nbsp; germline&nbsp; mutations in&nbsp; BRCA1&nbsp; were&nbsp; identified&nbsp; in&nbsp; individuals with a family history of breast and/or ovarian&nbsp; cancer&nbsp; (Castilla&nbsp; et&nbsp; al.&nbsp; 1994; Friedman et al. 1994; Miki et al. 1994). In addition to an elevated risk of breast and ovarian cancer, BRCA1 mutation carriers were&nbsp; also&nbsp; found&nbsp; to&nbsp; have&nbsp; an&nbsp; increased risk&nbsp; of&nbsp; colon,&nbsp; pancreatic,&nbsp; endometrial, cervical and prostate cancer (Brose et al. 2002;&nbsp; Ford&nbsp; et&nbsp; al.&nbsp; 1994;&nbsp; Struewing&nbsp; et&nbsp; al. 1997; Thompson et al. 2002b; Warner et al. 1999). However, the risk of cancer at these sites is relatively small&nbsp; when compared&nbsp; to&nbsp; the&nbsp; risk&nbsp; of&nbsp; cancer&nbsp; in&nbsp; the&nbsp; breast or&nbsp; ovary.&nbsp; Direct&nbsp; evidence&nbsp; that&nbsp; BRCA1 acted&nbsp; as&nbsp; a&nbsp; tumour&nbsp; suppressor&nbsp; in&nbsp; human breast&nbsp; and&nbsp; ovarian&nbsp; cancer&nbsp; cells&nbsp; came from&nbsp; in&nbsp; vitro&nbsp; studies&nbsp; where&nbsp; its&nbsp; over-expression&nbsp; retarded&nbsp; cell&nbsp; growth&nbsp; and&nbsp; its&nbsp; in- hibition&nbsp; accelerated&nbsp; cell&nbsp; growth,&nbsp; while mutated&nbsp; forms&nbsp; of&nbsp; the&nbsp; protein&nbsp; had&nbsp; no&nbsp; effect&nbsp; (Holt&nbsp; et&nbsp; al.&nbsp; 1996;&nbsp; Thompson&nbsp; et&nbsp; al. 1995). A second breast cancer susceptibility&nbsp; gene,&nbsp; BRCA2&nbsp; was&nbsp; subsequently identified in 1994 (Wooster et al. 1994).</p>\r\n<p>\r\n	The&nbsp; histopathology&nbsp; of&nbsp; breast&nbsp; cancers diagnosed&nbsp; in&nbsp; BRCA1&nbsp; mutation&nbsp; carriers differ&nbsp; substantially&nbsp; from&nbsp; those&nbsp; that&nbsp; occur in&nbsp; sporadic&nbsp; disease.&nbsp; Breast&nbsp; cancers&nbsp; that develop&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers&nbsp; are diagnosed at&nbsp; younger&nbsp; age, are of higher grade, often exhibit higher mitotic counts and&nbsp; have&nbsp; a greater&nbsp; proportion&nbsp; of&nbsp; tumour with&nbsp; continuous&nbsp; pushing&nbsp; margin&nbsp; (Breast Cancer Linkage Consortium 1997; Eerola et&nbsp; al.&nbsp; 2005;&nbsp; Eisinger&nbsp; et al.&nbsp; 1996; Jacquemier&nbsp; et al. 1995;&nbsp; Lakhani&nbsp; et al. 1998;&nbsp; Marcus&nbsp; et al. 1996;&nbsp; Moller&nbsp; et al. 2002; Querzoli et al. 2001; Robson et al. 1998;&nbsp; Stoppa-Lyonnet&nbsp; et al. 2000). Breast tumours in BRCA1 mutation carri- ers are more likely to be negative for ER, PR&nbsp; and&nbsp; AR&nbsp; when&nbsp; compared&nbsp; to sporadic breast cancers (Berns et al. 2003; Eerola et al. 2005; Foulkes et al. 2004; Lakhani et al. 2002; Loman et al. 1998; Osin et al. 1998;&nbsp; Palacios&nbsp; et al. 2005;&nbsp; Vaziri&nbsp; et al. 2001; Verhoog et al. 1999). They contain less&nbsp; tubule&nbsp; formation,&nbsp; more&nbsp; nuclear&nbsp; polymorphism&nbsp; and more&nbsp; lymphocytic&nbsp; infiltrate compared&nbsp; with their sporadic counterparts (Breast Cancer Linkage Consortium&nbsp; 1997; Eisinger&nbsp; et al. 1996;&nbsp; Lakhani et al. 1998). In addition, BRCA1 mutation carriers&nbsp; are&nbsp; more&nbsp; likely&nbsp; to&nbsp; develop&nbsp; contralateral&nbsp; breast&nbsp; cancer&nbsp; (Robson&nbsp; et al. 1998; Verhoog et al. 1999). Premalignant lesions&nbsp; associated&nbsp; with&nbsp; increased&nbsp; malig- nancy,&nbsp; such&nbsp; as&nbsp; ductal&nbsp; carcinoma&nbsp; in situ (DCIS),&nbsp; atypical&nbsp; ductal&nbsp; hyperplasia (ADH),&nbsp; and atypical&nbsp; lobular&nbsp; carcinoma (ALH),&nbsp; may&nbsp; be more&nbsp; prevalent&nbsp; in the normal&nbsp; breast&nbsp; tissue&nbsp; from&nbsp; BRCA1&nbsp; muta- tion&nbsp; carriers&nbsp; compared&nbsp; to the breast&nbsp; of patients&nbsp; without&nbsp; a known&nbsp; genetic&nbsp; predisposition to the disease (Hoogerbrugge et al.&nbsp; 2003;&nbsp; Kauff&nbsp; et al.&nbsp; 2003;&nbsp; Russo&nbsp; et&nbsp; al. 2001;&nbsp; Sun&nbsp; et al.&nbsp; 1996).&nbsp; Thus,&nbsp; women with an inherited predisposition to breast cancer&nbsp; due to a BRCA1&nbsp; mutation&nbsp; often present&nbsp; clinically&nbsp; with&nbsp; breast&nbsp; tumours containing&nbsp; features&nbsp; associated&nbsp; with&nbsp; a highly proliferative&nbsp; phenotype and&nbsp; with a worse prognosis.</p>\r\n<p>\r\n	To date, more than 1600 distinct pathogenic&nbsp; mutations,&nbsp; polymorphisms&nbsp; and variants&nbsp; in&nbsp; the&nbsp; BRCA1&nbsp; gene&nbsp; have&nbsp; been identified.&nbsp; In addition,&nbsp; over&nbsp; 1800 distinct pathogenic&nbsp; mutations&nbsp; have&nbsp; been identified&nbsp; in BRCA2&nbsp; (Breast&nbsp; Cancer&nbsp; Information&nbsp; Core&nbsp; 2005).&nbsp; The&nbsp; mutations&nbsp; are scattered&nbsp; throughout&nbsp; the&nbsp; entire&nbsp; coding region&nbsp; of&nbsp; the genes&nbsp; and&nbsp; show&nbsp; no clus- tering&nbsp; or&nbsp; &ldquo;hot&nbsp; spots&rdquo;.&nbsp; The&nbsp; frequency&nbsp; of these&nbsp; mutations&nbsp; varies&nbsp; widely&nbsp; among different populations and is influenced by founder effects. For example, in the Ashkenazi Jewish community four mutations are thought to account for the majority of inherited&nbsp; breast&nbsp; and/or&nbsp; ovarian&nbsp; cancer: 185delAG, 188del11 and 5382insC in the BRCA1&nbsp; gene&nbsp; and&nbsp; 6174delT&nbsp; in&nbsp; the BRCA2 gene (Tonin et al. 1996). In original&nbsp; analyses&nbsp; by&nbsp; the&nbsp; Breast&nbsp; Cancer&nbsp; Linkage&nbsp; Consortium&nbsp; (BCLC),&nbsp; BRCA1&nbsp; muta- tions&nbsp; were&nbsp; shown&nbsp; to&nbsp; occur&nbsp; in&nbsp; &gt;90%&nbsp; of families&nbsp; that&nbsp; contained&nbsp; at&nbsp; least&nbsp; four breast and/or ovarian cancers; and were estimated&nbsp; to&nbsp; occur&nbsp; in&nbsp; 45%&nbsp; of&nbsp; sporadic cases&nbsp; (Easton&nbsp; et&nbsp; al.&nbsp; 1993;&nbsp; Narod&nbsp; et&nbsp; al. 1995). However, since the cloning of this gene&nbsp; (Miki&nbsp; et&nbsp; al.&nbsp; 1994),&nbsp; the&nbsp; prevalence estimates&nbsp; for&nbsp; BRCA1&nbsp; have&nbsp; been&nbsp; much less&nbsp; than&nbsp; those&nbsp; estimated&nbsp; by&nbsp; initial&nbsp; linkage studies.&nbsp;</p>\r\n<p>\r\n	In&nbsp; hereditary&nbsp; breast&nbsp; cancer,&nbsp; mutations in&nbsp; BRCA1&nbsp; have&nbsp; been&nbsp; shown&nbsp; to&nbsp; occur&nbsp; in the&nbsp; large&nbsp; proportion&nbsp; of&nbsp; cases&nbsp; (~30-80%) in&nbsp; families&nbsp; with&nbsp; both&nbsp; breast&nbsp; and&nbsp; ovarian cancer,&nbsp; whereas&nbsp; BRCA2&nbsp; mutations&nbsp; ac- count for a smaller proportion (~15-20%) (de la Hoya et al. 2002; Diez et al. 2003; Ford et al. 1998; German Consortium for Hereditary&nbsp; Breast&nbsp; and&nbsp; Ovarian&nbsp; Cancer 2002;&nbsp; Martin&nbsp; et&nbsp; al.&nbsp; 2001;&nbsp; Narod&nbsp; et&nbsp; al. 1995;&nbsp; Wagner&nbsp; et&nbsp; al.&nbsp; 1998).&nbsp; In&nbsp; families containing&nbsp; breast&nbsp; cancer&nbsp; only,&nbsp; BRCA1 mutations&nbsp; occur&nbsp; in&nbsp; around&nbsp; 7%&nbsp; of&nbsp; cases where there is more than one first degree relative and in 28% of cases where there are&nbsp; four&nbsp; or&nbsp; more&nbsp; affected&nbsp; individuals within a family (Ford&nbsp; et al. 1998; Malone et al. 1998). BRCA1 mutations are more prevalent&nbsp; in&nbsp; ovarian&nbsp; cancer&nbsp; only&nbsp; families (compared to breast cancer only families) and&nbsp; are&nbsp; detected&nbsp; in&nbsp; 31-38%&nbsp; of&nbsp; cases (Antoniou&nbsp; et&nbsp; al.&nbsp; 2000;&nbsp; Gayther&nbsp; et&nbsp; al. 1999;&nbsp; Reedy&nbsp; et&nbsp; al.&nbsp; 2002).&nbsp; The&nbsp; prevalence of BRCA1 mutations in population- based studies is considerably lower than those&nbsp; estimated&nbsp; for&nbsp; large&nbsp; multiple-case families.&nbsp; Perhaps&nbsp; the&nbsp; most&nbsp; reliable&nbsp; estimate&nbsp; to&nbsp; date&nbsp; comes&nbsp; from&nbsp; a&nbsp; study&nbsp; conducted&nbsp; by&nbsp; Antoniou&nbsp; and&nbsp; colleagues, where&nbsp; population-based&nbsp; cohorts&nbsp; from&nbsp; 22 previous studies in 13 different countries were&nbsp; analysed.&nbsp; From&nbsp; 8139&nbsp; index&nbsp; cases presenting with breast or ovarian cancer, 3.5%&nbsp; were&nbsp; found&nbsp; to&nbsp; carry&nbsp; a&nbsp; mutation&nbsp; in BRCA1&nbsp; and&nbsp; 2.7%&nbsp; carried&nbsp; a&nbsp; mutation&nbsp; in BRCA2 (Antoniou et al. 2003). Moreover, breast&nbsp; cancer&nbsp; incidences&nbsp; were&nbsp; shown&nbsp; to increase with age, up to age 45-49 years in BRCA1-mutation carriers, but reached a&nbsp; plateau&nbsp; thereafter,&nbsp; whereas&nbsp; breast cancer&nbsp; incidence&nbsp; BRCA2-mutation&nbsp; carriers&nbsp; increased&nbsp; gradually&nbsp; with&nbsp; age (Antoniou&nbsp; et al.&nbsp; 2000;&nbsp; Brose et&nbsp; al. 2002; Scott et al. 2003).&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	Although&nbsp; BRCA1&nbsp; and&nbsp; 2 mutations&nbsp; are found&nbsp; in a high&nbsp; proportion&nbsp; of&nbsp; familial breast/ovarian&nbsp; cancer&nbsp; patients,&nbsp; defining the&nbsp; penetrance&nbsp; (the&nbsp; risk&nbsp; that&nbsp; a&nbsp; given mutation&nbsp; will&nbsp; lead&nbsp; to cancer)&nbsp; of&nbsp; BRCA1 mutations&nbsp; remains&nbsp; a challenging&nbsp; task. Early studies of large multiple-case families&nbsp; suggested&nbsp; that the lifetime&nbsp; risk&nbsp; of disease&nbsp; may&nbsp; be&nbsp; as&nbsp; high&nbsp; as&nbsp; 85%&nbsp; in BRCA1&nbsp; or&nbsp; BRCA2&nbsp; mutation&nbsp; carriers (Easton&nbsp; et&nbsp; al.&nbsp; 1993;&nbsp; Ford&nbsp; et al. 1994; Ford&nbsp; et al. 1998;&nbsp; Narod&nbsp; et al. 1995). However,&nbsp; more&nbsp; recent&nbsp; analyses&nbsp; have shown significantly lower risks than those initially&nbsp; estimated,&nbsp; 65%&nbsp; for&nbsp; the risk&nbsp; of breast&nbsp; cancer&nbsp; development&nbsp; and&nbsp; 39%&nbsp; for the&nbsp; risk&nbsp; of&nbsp; ovarian&nbsp; cancer&nbsp; development up&nbsp; to 70 years&nbsp; of&nbsp; age for&nbsp; BRCA1&nbsp; mutation carriers (Antoniou et al. 2000; Brose et&nbsp; al. 2002;&nbsp; Scott&nbsp; et al. 2003).&nbsp; The&nbsp; risk estimates&nbsp; for&nbsp; developing&nbsp; breast&nbsp; or&nbsp; ova- rian&nbsp; cancer&nbsp; for&nbsp; BRCA2&nbsp; mutation&nbsp; carriers are&nbsp; lower&nbsp; than&nbsp; that of&nbsp; BRCA1&nbsp; mutation carriers&nbsp; at 45%&nbsp; (breast)&nbsp; and&nbsp; 11%&nbsp; (ova- rian)&nbsp; respectively&nbsp; (Antoniou&nbsp; et al. 2000; Brose&nbsp; et&nbsp; al.&nbsp; 2002;&nbsp; Scott&nbsp; et al.&nbsp; 2003). Nevertheless,&nbsp; regardless&nbsp; of&nbsp; the precise penetrance&nbsp; estimate,&nbsp; BRCA1&nbsp; and&nbsp; 2 are highly&nbsp; penetrant&nbsp; (albeit&nbsp; not&nbsp; completely) breast cancer susceptibility genes whose mutation&nbsp; significantly&nbsp; increases&nbsp; the risk of developing breast cancer.</p>\r\n<p>\r\n	<em>Does&nbsp; BRCA1&nbsp; mutation&nbsp; position&nbsp; alter disease phenotype?&nbsp; </em></p>\r\n<p>\r\n	A&nbsp; critical&nbsp; question&nbsp; in&nbsp; mutation&nbsp; screening of&nbsp; BRCA1&nbsp; is&nbsp; whether&nbsp; the&nbsp; position&nbsp; of&nbsp; the mutation&nbsp; correlates&nbsp; with&nbsp; the&nbsp; cancer&nbsp; phenotype&nbsp; (i.e.&nbsp; breast&nbsp; or&nbsp; ovarian&nbsp; cancer). Knowing the biological consequence of a particular&nbsp; mutation&nbsp; could&nbsp; provide&nbsp; valuable information to patients seeking coun- selling on which type of preventative and management measures to undertake, if a mutation&nbsp; is&nbsp; found.&nbsp; The&nbsp; first&nbsp; formal&nbsp; evidence&nbsp; linking&nbsp; risk&nbsp; to&nbsp; mutation&nbsp; position came&nbsp; from&nbsp; Gayther&nbsp; et&nbsp; al.,&nbsp; (1995)&nbsp; who found that for 22 different mutations in 32 families,&nbsp; the&nbsp; ratio&nbsp; of&nbsp; breast&nbsp; to&nbsp; ovarian cancer&nbsp; was&nbsp; significantly&nbsp; lower&nbsp; after&nbsp; a &ldquo;change&nbsp; point&rdquo;.&nbsp; This&nbsp; change&nbsp; point&nbsp; was codon 1435 in exon 13, where a BRCA1 mutation 5&rsquo; of this codon confered a significantly&nbsp; higher&nbsp; risk&nbsp; of&nbsp; developing&nbsp; ova- rian&nbsp; cancer&nbsp; (Gayther&nbsp; et&nbsp; al.&nbsp; 1995).&nbsp; This has&nbsp; been&nbsp; confirmed&nbsp; by&nbsp; a&nbsp; study&nbsp; conducted&nbsp; in&nbsp; the&nbsp; UK&nbsp; (The&nbsp; Scottish/Northern Irish&nbsp; BRCA1/BRCA2&nbsp; Consortium&nbsp; 2003), however,&nbsp; other&nbsp; studies&nbsp; have&nbsp; failed&nbsp; to confirm&nbsp; a&nbsp; relationship&nbsp; between&nbsp; mutation position and cancer phenotype (Frank et al. 1998; Serova et al. 1996).&nbsp;</p>\r\n<p>\r\n	In&nbsp; a&nbsp; population-based&nbsp; study&nbsp; of&nbsp; women with&nbsp; BRCA1-related&nbsp; ovarian&nbsp; cancer,&nbsp; the risk of breast cancer in first degree relatives was found to increase proportionally as&nbsp; the&nbsp; mutation&nbsp; position&nbsp; approached&nbsp; the carboxyl&nbsp; terminus&nbsp; of&nbsp; the&nbsp; gene&nbsp; (Risch&nbsp; et al. 2001). In contrast to previous reports, a study conducted by&nbsp; the&nbsp; Breast Cancer Linage&nbsp; Consortium&nbsp; of&nbsp; 356&nbsp; BRCA1-related families found that mutations in the central&nbsp; portion&nbsp; of&nbsp; the&nbsp; gene&nbsp; (nucleotides 2401-4190)&nbsp; had&nbsp; a&nbsp; significantly&nbsp; higher ovarian:&nbsp; breast&nbsp; cancer&nbsp; ratio&nbsp; when&nbsp; com- pared&nbsp; to&nbsp; mutations&nbsp; between&nbsp; nucleotides 1-2400&nbsp; or&nbsp; nucleotides&nbsp; 4191-end (Thompson&nbsp; et&nbsp; al.&nbsp; 2002a).&nbsp; This&nbsp; was&nbsp; supported&nbsp; by&nbsp; an&nbsp; Australian&nbsp; study&nbsp; which,&nbsp; al- though subjects with ovarian cancer were not&nbsp; included, found that mutations&nbsp; within the&nbsp; central&nbsp; portion&nbsp; of&nbsp; the&nbsp; gene&nbsp; (2356- 4592) were associated with a decreased risk of breast cancer, compared to muta- tions&nbsp; at the&nbsp; N-&nbsp; and C-terminus&nbsp; (Scott&nbsp; et al.&nbsp; 2003).&nbsp; Furthermore,&nbsp; the same&nbsp; geno- type-phenotype&nbsp; relationship&nbsp; was&nbsp; also observed&nbsp; in&nbsp; a meta-analysis&nbsp; of&nbsp; 22 previous&nbsp; studies,&nbsp; although&nbsp; statistical&nbsp; significance was not achieved (Antoniou et al. 2003).&nbsp;</p>\r\n<p>\r\n	Although&nbsp; it is&nbsp; possible&nbsp; that&nbsp; the position of&nbsp; the mutation&nbsp; within&nbsp; BRCA1&nbsp; will&nbsp; affect the&nbsp; prevalence&nbsp; of&nbsp; breast&nbsp; or&nbsp; ovarian&nbsp; cancer, there is no solid&nbsp; evidence as&nbsp; yet, to suggest that this is the case. The available research is limited and has produced conflicting&nbsp; results&nbsp; as&nbsp; to which&nbsp; mutated regions present greater risk for breast or ovarian cancer.&nbsp;</p>\r\n<p>\r\n	<em>Other&nbsp; high&nbsp; penetrance&nbsp; breast&nbsp; cancer susceptibility genes&nbsp; </em></p>\r\n<p>\r\n	High penetrance breast cancer suscepti- bility&nbsp; genes,&nbsp; other&nbsp; than&nbsp; BRCA1/2,&nbsp; have been&nbsp; identified&nbsp; as&nbsp; a&nbsp; result&nbsp; of&nbsp; their&nbsp; association&nbsp; with&nbsp; other&nbsp; inherited&nbsp; cancer&nbsp; syn- dromes&nbsp; and&nbsp; include&nbsp; TP53,&nbsp; PTEN&nbsp; and STK11.&nbsp;</p>\r\n<p>\r\n	The&nbsp; p53 tumour&nbsp; suppressor&nbsp; protein&nbsp; is essential&nbsp; for&nbsp; the&nbsp; response&nbsp; to DNA&nbsp; dam- age and is classed as a &ldquo;guardian of the genome&rdquo;&nbsp; (Rozan&nbsp; &amp; El-Deiry&nbsp; 2007; Vousden &amp; Lu 2002). The involvement of the&nbsp; p53&nbsp; protein,&nbsp; encoded&nbsp; by&nbsp; TP53,&nbsp; in multiple cellular pathways&nbsp; involved in tumour&nbsp; suppression&nbsp; has&nbsp; long&nbsp; been&nbsp; established,&nbsp; and&nbsp; somatic&nbsp; mutations&nbsp; in the gene&nbsp; are&nbsp; frequently&nbsp; observed&nbsp; in many types&nbsp; of&nbsp; solid&nbsp; tumours&nbsp; (Olivier&nbsp; et al. 2009).&nbsp; Germline&nbsp; mutations&nbsp; in&nbsp; TP53 cause&nbsp; Li-Fraumeni&nbsp; syndrome,&nbsp; an inhe- rited&nbsp; disorder&nbsp; that&nbsp; greatly&nbsp; increases&nbsp; susceptibility&nbsp; to breast&nbsp; and&nbsp; other&nbsp; cancers. Although if present they are highly penetrant, conferring up to a 10 fold increase in&nbsp; the&nbsp; risk&nbsp; of&nbsp; developing&nbsp; breast&nbsp; cancer, these&nbsp; mutations&nbsp; are&nbsp; very&nbsp; rare&nbsp; and account&nbsp; for&nbsp; a much&nbsp; lower&nbsp; familial&nbsp; risk&nbsp; than BRCA1&nbsp; and BRCA2 (Lalloo et al. 2006). Moreover, they are uncommon in non-Li-Fraumeni&nbsp; breast&nbsp; cancer&nbsp; families (Borresen 1992; Lalloo et al. 2006).&nbsp;&nbsp;</p>\r\n<p>\r\n	Germline&nbsp; mutations&nbsp; in&nbsp; PTEN&nbsp; and STK11 are known to lead to Cowden and Peutz-Jeghers&nbsp; syndrome&nbsp; respectively (Hearle&nbsp; et&nbsp; al.&nbsp; 2006b;&nbsp; Nelen&nbsp; et&nbsp; al.&nbsp; 1996). Individuals&nbsp; with&nbsp; Cowden&nbsp; syndrome&nbsp; have an&nbsp; estimated&nbsp; 30-50%&nbsp; risk&nbsp; of&nbsp; developing breast&nbsp; cancer&nbsp; by&nbsp; the&nbsp; age&nbsp; of&nbsp; 70&nbsp; years (Nelen&nbsp; et&nbsp; al.&nbsp; 1996),&nbsp; while&nbsp; the&nbsp; risk&nbsp; of&nbsp; de- veloping&nbsp; breast&nbsp; cancer&nbsp; is&nbsp; approximately 45%&nbsp; in&nbsp; individuals&nbsp; with&nbsp; Peutz-Jeghers syndrome (Hearle et al. 2006a; Lim et al. 2004).&nbsp; However,&nbsp; PTEN&nbsp; and&nbsp; STK11&nbsp; mutations&nbsp; are&nbsp; rare&nbsp; in&nbsp; high&nbsp; risk breast/ovarian&nbsp; cancer&nbsp; individuals&nbsp; that&nbsp; do not&nbsp; carry&nbsp; BRCA1/2&nbsp; mutations&nbsp; (Guenard et&nbsp; al.&nbsp; 2007;&nbsp; Guenard&nbsp; et&nbsp; al.&nbsp; 2010).&nbsp; Interestingly,&nbsp; gross&nbsp; aberrations&nbsp; in&nbsp; PTEN were&nbsp; found&nbsp; in&nbsp; BRCA1&nbsp; heterozygous mutation&nbsp; carriers&nbsp; and&nbsp; loss&nbsp; of&nbsp; PTEN&nbsp; expression&nbsp; was&nbsp; associated&nbsp; with&nbsp; basal-like sporadic&nbsp; breast&nbsp; cancer&nbsp; cases,&nbsp; indicating that its loss may be tightly linked with deficiencies&nbsp; in&nbsp; the&nbsp; double-strand&nbsp; break DNA repair pathway (Saal et al. 2008).&nbsp;&nbsp;</p>\r\n<p>\r\n	Together, mutations in BRCA1/2, TP53, PTEN and STK11, although highly penetrant, only account for around 20-25% of the familial risk of breast cancer.&nbsp; Moreo- ver,&nbsp; mutations&nbsp; in&nbsp; TP53,&nbsp; PTEN&nbsp; and STK11&nbsp; are&nbsp; rare&nbsp; in&nbsp; patients&nbsp; who&nbsp; do&nbsp; not have&nbsp; the&nbsp; cancer&nbsp; syndromes&nbsp; associated with these genes and they are unlikely to contribute&nbsp; to&nbsp; a&nbsp; substantial&nbsp; fraction&nbsp; of breast&nbsp; cancer&nbsp; susceptibility&nbsp; in&nbsp; high-risk breast&nbsp; cancer&nbsp; families&nbsp; who&nbsp; do&nbsp; not&nbsp; harbour&nbsp; inactivating&nbsp; mutations&nbsp; in&nbsp; the BRCA1/2 tumour suppressor genes.</p>\r\n<p>\r\n	<strong>VARIANTS&nbsp; IN&nbsp; BREAST&nbsp; CANCER SUSCEPTIBILITY&nbsp; GENES&nbsp; WITH INTERMEDIATE PENETRANCE</strong></p>\r\n<p>\r\n	Four&nbsp; intermediate-penetrance&nbsp; breast cancer&nbsp; susceptibility&nbsp; genes,&nbsp; CHEK2, ATM, PALB2, and BRIP1, were identified through&nbsp; direct&nbsp; re-sequencing&nbsp; of&nbsp; candidate&nbsp; genes&nbsp; in&nbsp; familial&nbsp; association&nbsp; studies. Disease-causing mutations in these genes are predominantly ones that cause premature protein truncation, but are rare and confer a relative risk of breast cancer of 2- to 4-fold. All four genes are involved in the BRCA1 DNA repair pathway.&nbsp;</p>\r\n<p>\r\n	<em>ATM and CHEK2 </em></p>\r\n<p>\r\n	BRCA1 is a target for phosphorylation by several&nbsp; checkpoint&nbsp; kinases&nbsp; in&nbsp; response to&nbsp; DNA&nbsp; damage.&nbsp; The&nbsp; kinases&nbsp; that&nbsp; participate&nbsp; in&nbsp; this&nbsp; signalling&nbsp; cascade&nbsp; up- stream&nbsp; of&nbsp; BRCA1&nbsp; include&nbsp; ATM&nbsp; and&nbsp; the checkpoint&nbsp; protein&nbsp; CHEK2.&nbsp; ATM&nbsp; is&nbsp; a protein&nbsp; kinase&nbsp; that&nbsp; is&nbsp; structurally&nbsp; related to&nbsp; the&nbsp; phosphoinositide-3&nbsp; kinase&nbsp; (PI3K)- related&nbsp; kinase&nbsp; family&nbsp; and&nbsp; is&nbsp; a&nbsp; central component&nbsp; of&nbsp; the&nbsp; DNA&nbsp; damage&nbsp; response&nbsp; (Yang&nbsp; et&nbsp; al.&nbsp; 2003;&nbsp; Zhou&nbsp; &amp; Elledge&nbsp; 2000).&nbsp; CHEK2&nbsp; is&nbsp; activated&nbsp; by ATM&nbsp; and&nbsp; the&nbsp; phosphorylation&nbsp; of&nbsp; BRCA1 by&nbsp; CHEK2&nbsp; has&nbsp; been&nbsp; shown&nbsp; to&nbsp; govern which&nbsp; pathway&nbsp; DNA&nbsp; damage&nbsp; will&nbsp; be&nbsp; re- paired&nbsp; by:&nbsp; phosphorylation&nbsp; of&nbsp; BRCA1&nbsp; at Ser-988 promotes the repair of damaged DNA&nbsp; by&nbsp; homologous&nbsp; recombination&nbsp; (error-free),&nbsp; while&nbsp; suppressing&nbsp; repair&nbsp; by non-homologous&nbsp; recombination&nbsp; (error prone)&nbsp; (Zhang&nbsp; et&nbsp; al.&nbsp; 2004).&nbsp; It&nbsp; was&nbsp; dem- onstrated&nbsp; by&nbsp; Renwick&nbsp; and&nbsp; colleagues that&nbsp; monoallelic&nbsp; ATM&nbsp; mutations&nbsp; were present in 2.04% of familial breast cancer cases&nbsp; and&nbsp; were&nbsp; associated&nbsp; with&nbsp; a&nbsp; 2.37 fold&nbsp; increased&nbsp; breast&nbsp; cancer&nbsp; risk (Renwick&nbsp; et&nbsp; al.&nbsp; 2006).&nbsp; The CHEK2*1100delC&nbsp; was&nbsp; found&nbsp; to&nbsp; have&nbsp; a frequency&nbsp; of&nbsp; 1.1%&nbsp; in&nbsp; healthy&nbsp; individuals and&nbsp; 5.1%&nbsp; in&nbsp; individuals&nbsp; with&nbsp; breast&nbsp; can-cer from 718 BRCA1 or BRCA2 mutation negative&nbsp; families&nbsp; (Meijers-Heijboer&nbsp; et&nbsp; al. 2002).&nbsp; In&nbsp; the&nbsp; largest&nbsp; study&nbsp; to&nbsp; date,&nbsp; in- volving&nbsp; over&nbsp; 26,000&nbsp; cases&nbsp; and&nbsp; 27,000 controls,&nbsp; heterozygosity&nbsp; for&nbsp; the CHEK2*1100delC was associated with a 3-5 fold increase in the risk of developing breast&nbsp; cancer.&nbsp; Furthermore,&nbsp; in&nbsp; familial breast&nbsp; cancer&nbsp; cases&nbsp; the CHEK2*1100delC was associated with a 37% lifetime risk of breast cancer, which was&nbsp; almost&nbsp; as&nbsp; high&nbsp; as&nbsp; the&nbsp; lifetime&nbsp; risks conferred by mutations in BRCA1 or 2 in this study (Weischer et al. 2008).&nbsp;</p>\r\n<p>\r\n	<em>BRIP1 and PALB2</em></p>\r\n<p>\r\n	BRIP1&nbsp; and&nbsp; PALB2&nbsp; encode&nbsp; for&nbsp; proteins that co-localize with BRCA1 and BRCA2 to&nbsp; mediate&nbsp; the&nbsp; repair&nbsp; of&nbsp; DNA&nbsp; double-strand&nbsp; breaks&nbsp; (De&nbsp; Nicolo&nbsp; et&nbsp; al.&nbsp; 2008; Rahman et al. 2007; Seal et al. 2006; Xia et al. 2006). Truncating mutations arising in these&nbsp; genes are rare,&nbsp; exhibited in ap- proximately&nbsp; 0.1% of the United&nbsp; Kingdom breast&nbsp; cancer&nbsp; cohort,&nbsp; and&nbsp; confer&nbsp; an&nbsp; ap- proximately&nbsp; two-fold&nbsp; increase&nbsp; in&nbsp; the&nbsp; risk of breast cancer compared to population risks&nbsp; (Rahman&nbsp; et&nbsp; al.&nbsp; 2007;&nbsp; Seal&nbsp; et&nbsp; al. 2006). Founder mutations have been ob- served&nbsp; in&nbsp; French-Canadian,&nbsp; Finnish&nbsp; and Polish populations, allowing for the iden- tification&nbsp; of&nbsp; a&nbsp; gene-disease&nbsp; association (Dansonka-Mieszkowska&nbsp; et&nbsp; al.&nbsp; 2010; Erkko&nbsp; et&nbsp; al.&nbsp; 2007;&nbsp; Foulkes&nbsp; et&nbsp; al.&nbsp; 2007). However,&nbsp; mutations&nbsp; in&nbsp; BRIP1&nbsp; and PALB2&nbsp; show&nbsp; incomplete&nbsp; segregation with disease in relatives of probands who have breast cancer (Rahman et al. 2007; Xia et al. 2007), making it difficult to use this information in predictive screening to estimate&nbsp; individual&nbsp; breast&nbsp; cancer&nbsp; risk. There&nbsp; is&nbsp; no&nbsp; evidence&nbsp; to&nbsp; date&nbsp; that&nbsp; mo- noallelic&nbsp; mutations&nbsp; in&nbsp; BRIP1&nbsp; and&nbsp; PALB2 confer&nbsp; a&nbsp; phenotype&nbsp; beyond&nbsp; predisposition&nbsp; to&nbsp; breast&nbsp; cancer,&nbsp; although&nbsp; biallelic mutations&nbsp; in&nbsp; these&nbsp; genes&nbsp; are&nbsp; implicated in Fanconi anemia (Reid et al. 2007).&nbsp;</p>\r\n<p>\r\n	Inactivating&nbsp; mutations&nbsp; in&nbsp; the&nbsp; intermediate&nbsp; penetrance&nbsp; genes&nbsp; ATM,&nbsp; CHEK2, BRIP1&nbsp; and&nbsp; PALB2&nbsp; are&nbsp; rare,&nbsp; with&nbsp; fewer than&nbsp; 1%&nbsp; of&nbsp; the&nbsp; population&nbsp; being&nbsp; heterozygote&nbsp; for&nbsp; these&nbsp; variants.&nbsp; Collectively, mutations in these genes account for approximately&nbsp; 2.3%&nbsp; of&nbsp; the&nbsp; familial&nbsp; breast cancer risk (Rahman et al. 2007).&nbsp;</p>\r\n<p>\r\n	<strong>THE&nbsp; IDENTIFICATION&nbsp; OF&nbsp; NEW&nbsp; LOW RISK VARIANTS&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	<em>Genetic&nbsp; modifiers&nbsp; of&nbsp; risk&nbsp; in&nbsp; the&nbsp; general population&nbsp;&nbsp;&nbsp; </em></p>\r\n<p>\r\n	Since the identification of BRCA1 twenty years&nbsp; ago&nbsp; (Hall&nbsp; et&nbsp; al.&nbsp; 1990;&nbsp; Miki&nbsp; et&nbsp; al. 1994), numerous studies have used candidate&nbsp; gene approaches to identify other breast&nbsp; cancer&nbsp; susceptibility&nbsp; genes (BRCAx)&nbsp; whose&nbsp; mutations&nbsp; may&nbsp; lead&nbsp; to an&nbsp; increased&nbsp; risk&nbsp; of&nbsp; breast&nbsp; cancer,&nbsp; with limited success- BRCAx still remains elusive. Mutations in the high and moderate penetrance&nbsp; breast&nbsp; cancer&nbsp; susceptibility genes&nbsp; described&nbsp; above&nbsp; account&nbsp; for&nbsp; ap- proximately&nbsp; 25%&nbsp; of&nbsp; breast&nbsp; cancers;&nbsp; are rarely&nbsp; found&nbsp; in&nbsp; sporadic&nbsp; cases&nbsp; and&nbsp; thus, much of the genetic component of breast cancer&nbsp; risk&nbsp; remains&nbsp; to&nbsp; be&nbsp; characterised. The&nbsp; majority&nbsp; of&nbsp; breast&nbsp; cancers&nbsp; do&nbsp; not arise&nbsp; through&nbsp; typical&nbsp; patterns&nbsp; of&nbsp; familial inheritance, they occur sporadically. Sporadic breast cancers differ markedly from familial&nbsp; breast&nbsp; cancers;&nbsp; with&nbsp; respect&nbsp; to their&nbsp; age&nbsp; of&nbsp; onset&nbsp; (post-menopause&nbsp; versus&nbsp; pre-menopause),&nbsp; their&nbsp; phenotype (receptor&nbsp; positive&nbsp; versus&nbsp; receptor&nbsp; nega- tive&nbsp; in&nbsp; BRCA1&nbsp; mutation&nbsp; carriers)&nbsp; and their&nbsp; clinical&nbsp; behaviour&nbsp; (familial&nbsp; breast cancers&nbsp; tend&nbsp; to&nbsp; be&nbsp; more&nbsp; aggressive). Thus,&nbsp; candidate&nbsp; gene&nbsp; approaches&nbsp; in large&nbsp; familial&nbsp; pedigree&nbsp; studies&nbsp; are&nbsp; likely to&nbsp; miss&nbsp; common&nbsp; genetic&nbsp; variation&nbsp; that confers&nbsp; a&nbsp; risk&nbsp; of&nbsp; breast&nbsp; cancer&nbsp; in&nbsp; the majority of the population.&nbsp; Genome-wide association studies (GWAS) in very large study populations of breast cancer cases and&nbsp; unaffected&nbsp; controls&nbsp; have&nbsp; gained momentum&nbsp; in&nbsp; recent&nbsp; years,&nbsp; enabling&nbsp; an unbiased&nbsp; investigation&nbsp; of&nbsp; genetic&nbsp; variation&nbsp; across&nbsp; the&nbsp; whole&nbsp; genome,&nbsp; as&nbsp; opposed&nbsp; to&nbsp; examination&nbsp; of&nbsp; selected&nbsp; va- riants&nbsp; in&nbsp; a few key&nbsp; genes,&nbsp; in&nbsp; an&nbsp; effort to discover new breast cancer susceptibility loci. To date, 13 breast cancer susceptibility loci have been discovered by these&nbsp; approaches (Table&nbsp; 1). None of the iden- tified variants are in the coding regions of genes,&nbsp; although&nbsp; some&nbsp; of&nbsp; the regions contain or are nearby known genes (e.g. FGFR2,&nbsp; MAP3K1,&nbsp; RAD51L)&nbsp; and five&nbsp; of the&nbsp; variants&nbsp; are&nbsp; located&nbsp; in&nbsp; non-genic&nbsp; regions&nbsp; (1p11.2,&nbsp; 2q35,&nbsp; 3p24,&nbsp; 5p12,&nbsp; 8q24) (Ahmed&nbsp; et al. 2009;&nbsp; Cox&nbsp; et al. 2007; Easton&nbsp; et&nbsp; al. 2007;&nbsp; Hunter&nbsp; et al. 2007; Milne&nbsp; et al.&nbsp; 2009;&nbsp; Stacey&nbsp; et al. 2008; Thomas&nbsp; et al. 2009).&nbsp; The&nbsp; individual&nbsp; variants&nbsp; confer&nbsp; only&nbsp; a modest&nbsp; increase&nbsp; in the&nbsp; risk&nbsp; of&nbsp; breast&nbsp; cancer,&nbsp; of&nbsp; up to approximately 1.65 fold.&nbsp;</p>\r\n<p>\r\n	Importantly,&nbsp; candidate&nbsp; gene&nbsp; approaches&nbsp; and&nbsp; the&nbsp; recent&nbsp; GWA&nbsp; studies have&nbsp; found&nbsp; no&nbsp; evidence&nbsp; that&nbsp; common variants&nbsp; in&nbsp; high&nbsp; risk&nbsp; breast&nbsp; cancer&nbsp; sus- ceptibility&nbsp; genes&nbsp; such&nbsp; as&nbsp; BRCA1/2, TP53,&nbsp; ATM&nbsp; and&nbsp; CHEK2&nbsp; are&nbsp; associated with&nbsp; increased&nbsp; breast&nbsp; cancer&nbsp; risk (Baynes&nbsp; et&nbsp; al.&nbsp; 2007;&nbsp; Lu&nbsp; et&nbsp; al.&nbsp; 2010).&nbsp; In fact,&nbsp; few&nbsp; of&nbsp; the&nbsp; genes&nbsp; in&nbsp; the&nbsp; regions identified by GWAS had previously been reported&nbsp; to&nbsp; be&nbsp; associated&nbsp; with&nbsp; breast cancer.&nbsp; The&nbsp; strongest&nbsp; association&nbsp; with breast&nbsp; cancer&nbsp; risk&nbsp; was&nbsp; found&nbsp; for&nbsp; a&nbsp; SNP located&nbsp; in&nbsp; intron&nbsp; 2&nbsp; of&nbsp; FGFR2 (rs2981582),&nbsp; where&nbsp; women&nbsp; carrying&nbsp; two copies&nbsp; of&nbsp; the&nbsp; high&nbsp; risk&nbsp; allele&nbsp; were&nbsp; observed to have a 1.63 fold increased risk when&nbsp; compared&nbsp; to&nbsp; women&nbsp; who&nbsp; carried two&nbsp; copies&nbsp; of&nbsp; the&nbsp; low&nbsp; risk&nbsp; allele&nbsp; (Easton et al. 2007). The FGF signalling pathway has&nbsp; previously&nbsp; been&nbsp; shown&nbsp; to&nbsp; induce mammary&nbsp; tumours&nbsp; in&nbsp; mouse&nbsp; models (Dickson&nbsp; et&nbsp; al.&nbsp; 2000)&nbsp; and&nbsp; in&nbsp; particular, amplification&nbsp; of&nbsp; FGFR2&nbsp; has&nbsp; been&nbsp; shown to&nbsp; occur&nbsp; in&nbsp; 5-10%&nbsp; of&nbsp; human&nbsp; breast&nbsp; cancers&nbsp; (Adnane&nbsp; et&nbsp; al.&nbsp; 1991;&nbsp; Heiskanen&nbsp; et al. 2001). Recent studies by Meyer et al., have demonstrated that homozygotes for the&nbsp; high&nbsp; risk&nbsp; FGFR2&nbsp; allele&nbsp; have&nbsp; increased&nbsp; expression&nbsp; of&nbsp; FGFR2&nbsp; and&nbsp; func- tional&nbsp; studies&nbsp; have&nbsp; indicated&nbsp; that&nbsp; this&nbsp; is due&nbsp; to&nbsp; altered&nbsp; binding&nbsp; affinity&nbsp; of&nbsp; FGFR2 for&nbsp; the&nbsp; transcription&nbsp; factors&nbsp; Oct-1&nbsp; and C/EBP&nbsp; (Meyer&nbsp; et&nbsp; al.&nbsp; 2008).&nbsp; This&nbsp; was the&nbsp; first&nbsp; study&nbsp; to&nbsp; determine&nbsp; a&nbsp; biological consequence&nbsp; caused&nbsp; by&nbsp; variants&nbsp; identi- fied&nbsp; through&nbsp; GWA&nbsp; studies&nbsp; and clearly demonstrates&nbsp; a mechanism&nbsp; that may lead&nbsp; to increased&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in individuals&nbsp; who&nbsp; are&nbsp; homozygous&nbsp; for&nbsp; the high risk FGFR2 allele. However, a func- tional&nbsp; consequence&nbsp; for&nbsp; many&nbsp; of&nbsp; the&nbsp; variants&nbsp; still&nbsp; remains&nbsp; at large&nbsp; and the&nbsp; bio- logical&nbsp; mechanisms&nbsp; underlying&nbsp; the observed increase in breast cancer risk are unknown.</p>\r\n<p>\r\n	It&nbsp; is&nbsp; apparent&nbsp; that each&nbsp; individual&nbsp; va- riant identified through GWA studies only accounts&nbsp; for&nbsp; a modest&nbsp; increase&nbsp; in the risk&nbsp; of&nbsp; developing&nbsp; breast&nbsp; cancer,&nbsp; suggesting&nbsp; that&nbsp; most&nbsp; of&nbsp; the unexplained breast cancer risk could be accounted for by&nbsp; a polygenic&nbsp; model,&nbsp; where&nbsp; risk&nbsp; alleles act multiplicatively (Pharoah et al. 2008). A&nbsp; recent&nbsp; study&nbsp; by&nbsp; Reeves&nbsp; et al.,&nbsp; (2010) comparing&nbsp; 13&nbsp; risk&nbsp; alleles&nbsp; (Table&nbsp; 1 and ATM:&nbsp; rs1800054)&nbsp; in&nbsp; 10&nbsp; 306&nbsp; women&nbsp; with breast cancer and 10 393 women without breast cancer; has investigated the combined&nbsp; effect&nbsp; 7 SNPs&nbsp; that&nbsp; were&nbsp; most strongly and significantly associated with breast cancer risk individually (Reeves et al. 2010). Odds ratios (ORs) were shown to increase from 0.75 to 1.45 for cases in the&nbsp; bottom&nbsp; fifth&nbsp; quintile&nbsp; compared&nbsp; to cases&nbsp; in the&nbsp; top&nbsp; fifth&nbsp; quintile,&nbsp; however, ORs in the&nbsp; top fifth quintile did&nbsp; not differ greatly&nbsp; from&nbsp; the OR&nbsp; of&nbsp; the individual FGFR2&nbsp; allele&nbsp; (1.20),&nbsp; which&nbsp; showed&nbsp; the strongest&nbsp; association&nbsp; with&nbsp; breast&nbsp; cancer risk.&nbsp; The&nbsp; cumulative&nbsp; risk&nbsp; of&nbsp; developing breast cancer was 8.8% for cases in the top&nbsp; fifth&nbsp; quintile&nbsp; compared&nbsp; to 4.4%&nbsp; for cases&nbsp; in the bottom&nbsp; fifth&nbsp; quintile.&nbsp; Thus, although&nbsp; a polygenic&nbsp; risk&nbsp; model&nbsp; for&nbsp; the variants&nbsp; has&nbsp; been&nbsp; demonstrated,&nbsp; the cumulative&nbsp; lifetime&nbsp; risk&nbsp; (8.8%)&nbsp; of&nbsp; developing breast cancer in women in the top fifth&nbsp; quintile&nbsp; was&nbsp; shown&nbsp; to&nbsp; be similar&nbsp; to that of women with one affected first-degree relative (9.1%) and far less than that of&nbsp; women&nbsp; with&nbsp; two&nbsp; affected&nbsp; first-degree relatives (15.4%) (Reeves et al. 2010). In addition,&nbsp; it has&nbsp; been demonstrated&nbsp; that 12 of the identified variants show no inte- ractions&nbsp; with other risk factors for breast cancer&nbsp; including&nbsp; HRT,&nbsp; age&nbsp; at&nbsp; menopause, age at menarche, BMI and parity (Travis&nbsp; et&nbsp; al.&nbsp; 2010);&nbsp; and&nbsp; variants&nbsp; identified&nbsp; from&nbsp; GWA&nbsp; studies&nbsp; conducted&nbsp; by Hunter&nbsp; et&nbsp; al.,&nbsp; (Hunter&nbsp; et&nbsp; al.&nbsp; 2007)&nbsp; and Thomas et al. (Thomas et al. 2009) were found to have no association with breast cancer&nbsp; survival&nbsp; (Azzato&nbsp; et&nbsp; al.&nbsp; 2010). Thus at this stage, polygenic risk scores are not a useful tool to inform women of their risk of developing breast cancer and appear&nbsp; to&nbsp; have&nbsp; limited&nbsp; utility&nbsp; in&nbsp; breast cancer prognostication.&nbsp;</p>\r\n<p>\r\n	<em>Genetic&nbsp; modifiers&nbsp; of&nbsp; risk&nbsp; in&nbsp; BRCA mutation carriers&nbsp;&nbsp; </em></p>\r\n<p>\r\n	Given&nbsp; the&nbsp; substantial&nbsp; heterogeneity&nbsp; in penetrance&nbsp; estimates&nbsp; for&nbsp; BRCA1&nbsp; mutation&nbsp; carriers&nbsp; between&nbsp; studies&nbsp; of&nbsp; different populations, in addition to the variation in penetrance&nbsp; estimates&nbsp; observed&nbsp; between individuals,&nbsp; it&nbsp; is&nbsp; reasonable&nbsp; to&nbsp; postulate the existence of other factors which may modulate&nbsp; the&nbsp; risk&nbsp; of&nbsp; cancer&nbsp; in&nbsp; BRCA1 mutation carriers. In this way, &ldquo;modifiers&rdquo; may&nbsp; act&nbsp; in&nbsp; conjunction&nbsp; with&nbsp; the&nbsp; mutated BRCA1&nbsp; allele&nbsp; to&nbsp; bring&nbsp; about&nbsp; an&nbsp; earlier diagnosis&nbsp; in&nbsp; an&nbsp; individual,&nbsp; thus&nbsp; affecting the penetrance of this gene.&nbsp;</p>\r\n<p>\r\n	Candidate gene approaches have identified&nbsp; several&nbsp; genetic&nbsp; variants&nbsp; that&nbsp; may alter&nbsp; the&nbsp; risk&nbsp; of&nbsp; breast&nbsp; cancer&nbsp; in&nbsp; BRCA1 mutation&nbsp; carriers.&nbsp; There&nbsp; are&nbsp; several&nbsp; reports that allelic variants of the Androgen Receptor&nbsp; (AR)&nbsp; and&nbsp; the&nbsp; Amplified&nbsp; In Breast Cancer 1 (AIB1) gene, can modify the risk of breast cancer in BRCA1 mutation&nbsp; carriers&nbsp; (Kadouri&nbsp; et&nbsp; al.&nbsp; 2004; Rebbeck&nbsp; et&nbsp; al.&nbsp; 1999;&nbsp; Rebbeck&nbsp; et&nbsp; al. 2001).&nbsp; However,&nbsp; other&nbsp; studies&nbsp; have found&nbsp; no&nbsp; excess&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers&nbsp; according&nbsp; to their&nbsp; AR&nbsp; allele&nbsp; or&nbsp; AIB1&nbsp; polymorphism status (Hughes et al. 2005; Kadouri et al. 2001;&nbsp; Menin&nbsp; et&nbsp; al.&nbsp; 2001;&nbsp; Montgomery&nbsp; et al. 2005; Spurdle et al. 2005). TP53 may also play a role in BRCA1-related tumourigenesis.&nbsp; TP53&nbsp; has&nbsp; been&nbsp; found&nbsp; to be mutated&nbsp; in&nbsp; 53-77%&nbsp; of&nbsp; BRCA1-related breast&nbsp; cancers,&nbsp; but only&nbsp; in 30-35%&nbsp; of sporadic&nbsp; breast&nbsp; tumours&nbsp; (Crook&nbsp; et al. 1997; Crook et al. 1998; Greenblatt et al. 2001;&nbsp; Phillips&nbsp; et al.&nbsp; 1999;&nbsp; Sensi&nbsp; et al. 2003).&nbsp; Likewise,&nbsp; TP53&nbsp; mutations&nbsp; are found&nbsp; in 50-83%&nbsp; of&nbsp; ovarian&nbsp; cancers linked to BRCA1 compared to 30-49% of sporadic&nbsp; ovarian&nbsp; tumours&nbsp; (Buller&nbsp; et&nbsp; al. 2001;&nbsp; Ramus&nbsp; et al.&nbsp; 1999;&nbsp; Reedy&nbsp; et al. 2001;&nbsp; Rhei&nbsp; et al. 1998).&nbsp; Thus,&nbsp; the high rate of TP53 mutations found&nbsp; in cancers of BRCA1 mutation carriers indicates that inactivation of p53 function may act as a modifier&nbsp; of&nbsp; cancer&nbsp; risk&nbsp; in these&nbsp; women. However, subsequent studies have found no&nbsp; association&nbsp; between&nbsp; polymorphisms in&nbsp; TP53&nbsp; (Arg72Pro)&nbsp; or&nbsp; MDM&nbsp; (309G&gt;T) (either&nbsp; singly&nbsp; or&nbsp; in combination)&nbsp; and breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA1&nbsp; or&nbsp; BRCA2 mutation&nbsp; carriers,&nbsp; suggesting&nbsp; that&nbsp; the inactivation&nbsp; of&nbsp; TP53&nbsp; and&nbsp; its&nbsp; signalling pathway may occur as a result of tumour progression&nbsp; rather&nbsp; than being&nbsp; a risk modifier&nbsp; (Sinilnikova&nbsp; et al.&nbsp; 2009).&nbsp; The RAD51 135G&gt;C polymorphism has been shown&nbsp; to increase&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA2&nbsp; mutation&nbsp; carriers&nbsp; by&nbsp; approximately&nbsp; 3 fold,&nbsp; but&nbsp; does&nbsp; not increase breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA1&nbsp; mutation carriers&nbsp; or&nbsp; the general&nbsp; population (Antoniou et al.&nbsp; 2007; Zhou et al. 2010). Instead,&nbsp; the RAD51&nbsp; 135G&gt;C&nbsp; polymor- phism&nbsp; may&nbsp; protect&nbsp; against&nbsp; the&nbsp; risk&nbsp; of breast&nbsp; cancer&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers&nbsp; (Jakubowska&nbsp; et al.&nbsp; 2007c).&nbsp; There&nbsp; is also&nbsp; evidence&nbsp; that&nbsp; the&nbsp; 677C&gt;T&nbsp; polymorphism&nbsp; in Methylenetetrahydrofolate&nbsp; reductase (MTHFR) and the C to T transition&nbsp; in the&nbsp; 3&#39;&nbsp; untranslated&nbsp; region&nbsp; of&nbsp; the Prohibitin&nbsp; (PHB)&nbsp; may&nbsp; increase&nbsp; breast cancer&nbsp; risk&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers (Jakubowska&nbsp; et al. 2007a;&nbsp; Jakubowska et al.&nbsp; 2007b).&nbsp; Allele&nbsp; variants of the Insulin-like&nbsp; Growth&nbsp; Factor-1&nbsp; (IGF1)&nbsp; and&nbsp; the Progesterone&nbsp; Receptor&nbsp; (PR)&nbsp; are&nbsp; known to&nbsp; alter&nbsp; hormonal&nbsp; sensitivity&nbsp; and&nbsp; have been&nbsp; shown&nbsp; to&nbsp; modify&nbsp; the&nbsp; risk&nbsp; of&nbsp; breast and&nbsp; ovarian&nbsp; cancer&nbsp; respectively,&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers&nbsp; (Jernstrom&nbsp; et al.&nbsp; 2005;&nbsp; Runnebaum&nbsp; et&nbsp; al.&nbsp; 2001).&nbsp; Poly- morphisms in the Harvey Rat Sarcoma 1 (HRAS1)&nbsp; proto-oncogene&nbsp; as&nbsp; well&nbsp; as common variants of the BRCA1 wild-type allele itself have been shown to increase the&nbsp; risk&nbsp; of&nbsp; ovarian&nbsp; cancers&nbsp; in&nbsp; BRCA1 mutation&nbsp; carriers&nbsp; (Ginolhac&nbsp; et&nbsp; al.&nbsp; 2003; Phelan&nbsp; et&nbsp; al.&nbsp; 1996).&nbsp; Other&nbsp; groups&nbsp; have reported&nbsp; low&nbsp; expression&nbsp; of&nbsp; the&nbsp; pro- apoptotic&nbsp; protein&nbsp; B-cell&nbsp; Lymphoma&nbsp; Protein&nbsp; 2&nbsp; (Bcl-2)&nbsp; and&nbsp; amplification&nbsp; of&nbsp; the Myelocytomatosis&nbsp; (MYC)&nbsp; oncogene,&nbsp; in BRCA1-associated&nbsp; breast&nbsp; tumours&nbsp; com- pared to sporadic cases (Freneaux et al. 2000;&nbsp; Grushko&nbsp; et&nbsp; al.&nbsp; 2004),&nbsp; but&nbsp; whether these&nbsp; genes&nbsp; act&nbsp; as&nbsp; risk&nbsp; modifiers&nbsp; in BRCA-related&nbsp; tumourigenesis&nbsp; is&nbsp; unknown.&nbsp;</p>\r\n<p>\r\n	Numerous&nbsp; other&nbsp; candidate&nbsp; genes&nbsp; have been&nbsp; investigated&nbsp; as&nbsp; modifiers&nbsp; of BRCA1/2&nbsp; penetrance,&nbsp; however,&nbsp; they have&nbsp; been&nbsp; met&nbsp; with&nbsp; limited&nbsp; success. Given&nbsp; the&nbsp; lack&nbsp; of&nbsp; targets&nbsp; found&nbsp; to&nbsp; posi- tively correlate with breast cancer risk by candidate&nbsp; gene&nbsp; approaches&nbsp; in&nbsp; BRCA1/2 mutation carriers, GWAS studies are now being&nbsp; applied&nbsp; to&nbsp; BRCA1/2&nbsp; mutation&nbsp; car- riers.&nbsp; Initial&nbsp; studies&nbsp; have&nbsp; focussed&nbsp; on determining&nbsp; whether&nbsp; variants&nbsp; that&nbsp; increase&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in&nbsp; population based-studies&nbsp; confer&nbsp; a&nbsp; similar&nbsp; risk&nbsp; of breast&nbsp; cancer&nbsp; development&nbsp; in&nbsp; BRCA1/2 mutation carriers. To this end, five of the variants&nbsp; identified&nbsp; by&nbsp; GWAS&nbsp; (Table&nbsp; 1) have been genotyped in women who are BRCA1&nbsp; and&nbsp; 2&nbsp; mutation&nbsp; carriers.&nbsp; While variants&nbsp; in&nbsp; FGFR2,&nbsp; TNCR9,&nbsp; LSP1, MAP3K1&nbsp; and&nbsp; the&nbsp; 2q35&nbsp; region&nbsp; were&nbsp; as- sociated&nbsp; with&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA2&nbsp; mutation&nbsp; carriers,&nbsp; only&nbsp; the TNCR9 variant and the 2q35 region was associated&nbsp; with&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in BRCA1 mutation carriers (Antoniou et al. 2008;&nbsp; Antoniou&nbsp; et&nbsp; al.&nbsp; 2009).&nbsp; More&nbsp; recently,&nbsp; GWA&nbsp; studies&nbsp; have&nbsp; been&nbsp; carried out&nbsp; in&nbsp; BRCA1/2&nbsp; mutation&nbsp; carriers&nbsp; to identify&nbsp; new&nbsp; modifiers&nbsp; of&nbsp; disease&nbsp; risk&nbsp; in these&nbsp; individuals.&nbsp; Interestingly,&nbsp; FGFR2 was&nbsp; the only&nbsp; locus&nbsp; to reach&nbsp; GWAS&nbsp; sta- tistical significance and showed a similar magnitude of risk (approximately 1.3 fold increased&nbsp; risk)&nbsp; as&nbsp; reported&nbsp; for&nbsp; breast cancer&nbsp; risk&nbsp; in population-based&nbsp; studies (Gaudet&nbsp; et al.&nbsp; 2010).&nbsp; Thus,&nbsp; variants&nbsp; that modify&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; in the&nbsp; general population&nbsp; also&nbsp; appear&nbsp; to modify&nbsp; breast cancer&nbsp; risk&nbsp; in BRCA2&nbsp; mutation&nbsp; carriers. However, this does not appear to be the case&nbsp; in BRCA1&nbsp; mutation&nbsp; carriers&nbsp; where one&nbsp; locus&nbsp; (19p13)&nbsp; containing&nbsp; 5 variants has been reported to be associated&nbsp; with breast&nbsp; cancer&nbsp; risk&nbsp; in these&nbsp; individuals and&nbsp; a similar&nbsp; association&nbsp; was&nbsp; also&nbsp; reported&nbsp; for&nbsp; ER-negative&nbsp; and&nbsp; triple&nbsp; negative&nbsp; cases&nbsp; (Antoniou&nbsp; et al.&nbsp; 2010).&nbsp; These results seem to reflect the distinct biology of&nbsp; BRCA1-related&nbsp; breast&nbsp; cancers,&nbsp; which are&nbsp; predominantly&nbsp; hormone&nbsp; receptor negative,&nbsp; compared&nbsp; to BRCA2-related and sporadic breast cancers, and further highlights&nbsp; the&nbsp; genetically&nbsp; divergent&nbsp; path- ways that lead to tumourigenesis in these women.</p>\r\n<p>\r\n	<strong>SUMMARY AND CONCLUSIONS&nbsp; </strong></p>\r\n<p>\r\n	Following&nbsp; the&nbsp; identification&nbsp; of&nbsp; BRCA1 and&nbsp; BRCA2&nbsp; as&nbsp; high&nbsp; penetrance&nbsp; breast cancer&nbsp; susceptibility&nbsp; genes,&nbsp; significant effort&nbsp; has&nbsp; been&nbsp; aimed&nbsp; at&nbsp; identifying&nbsp; further&nbsp; susceptibility&nbsp; genes&nbsp; that&nbsp; may&nbsp; ac- count for the missing heritability to breast cancer.&nbsp; The&nbsp; advances&nbsp; made&nbsp; have&nbsp; been small and the elusive BRCAx has still not been&nbsp; found.&nbsp; This&nbsp; has&nbsp; led&nbsp; to&nbsp; the&nbsp; conclusion&nbsp; that&nbsp; there&nbsp; are&nbsp; in&nbsp; fact&nbsp; no&nbsp; other&nbsp; high penetrance&nbsp; breast&nbsp; cancer&nbsp; susceptibility genes&nbsp; and&nbsp; that&nbsp; the&nbsp; remainder&nbsp; of&nbsp; the&nbsp; genetic&nbsp; risk&nbsp; to&nbsp; breast&nbsp; cancer&nbsp; can&nbsp; be&nbsp; accounted for by a polygenic model, where many&nbsp; variants&nbsp; of&nbsp; low&nbsp; penetrance&nbsp; act multiplicatively&nbsp; to&nbsp; increase&nbsp; the&nbsp; relative risk&nbsp; of&nbsp; developing&nbsp; breast&nbsp; cancer.&nbsp; In&nbsp; the last 3 years, with considerable advances in&nbsp; technology,&nbsp; a&nbsp; multitude&nbsp; of&nbsp; data&nbsp; has been generated by GWAS leading to the identification&nbsp; of&nbsp; 13&nbsp; new&nbsp; low&nbsp; penetrance variants&nbsp; that&nbsp; individually&nbsp; account&nbsp; for&nbsp; a modest&nbsp; increase&nbsp; in&nbsp; breast&nbsp; cancer&nbsp; risk. However,&nbsp; the&nbsp; results&nbsp; have&nbsp; been&nbsp; somewhat&nbsp; disappointing,&nbsp; when&nbsp; combined these&nbsp; variants&nbsp; still&nbsp; only&nbsp; lead&nbsp; to&nbsp; a&nbsp; small increase&nbsp; in&nbsp; breast&nbsp; cancer&nbsp; risk&nbsp; and&nbsp; the information generated thus far, will not be informative&nbsp; in&nbsp; a&nbsp; clinical&nbsp; setting&nbsp; in&nbsp; determining&nbsp; an&nbsp; individuals&rsquo;&nbsp; risk&nbsp; of&nbsp; developing breast&nbsp; cancer&nbsp; and&nbsp; therefore,&nbsp; will&nbsp; not&nbsp; be useful in preventative strategies.</p>\r\n<p>\r\n	How far have we come from BRCA1 in predicting&nbsp; breast&nbsp; cancer&nbsp; risk?&nbsp; To&nbsp; date, most&nbsp; of&nbsp; the&nbsp; GWAS&nbsp; on&nbsp; breast&nbsp; cancer have been carried out in populations un- selected&nbsp; for&nbsp; ER&nbsp; status,&nbsp; grade,&nbsp; age,&nbsp; ethnicity,&nbsp; etc.&nbsp; But&nbsp; breast&nbsp; cancer&nbsp; is&nbsp; an&nbsp; extremely&nbsp; heterogeneous&nbsp; disease&nbsp; where gene-environment&nbsp; interactions,&nbsp; especially&nbsp; those&nbsp; influencing&nbsp; estrogen&nbsp; exposure,&nbsp; play&nbsp; a&nbsp; major&nbsp; role&nbsp; in&nbsp; determining disease&nbsp; onset&nbsp; and&nbsp; expression&nbsp; and&nbsp; this has&nbsp; been&nbsp; repeatedly&nbsp; demonstrated.&nbsp; In this&nbsp; regard,&nbsp; it&nbsp; has&nbsp; been&nbsp; shown&nbsp; that&nbsp; the relative&nbsp; risks&nbsp; for&nbsp; each&nbsp; of&nbsp; the&nbsp; 13&nbsp; low&nbsp; pe- netrance&nbsp; variants&nbsp; are&nbsp; significantly&nbsp; influ- enced&nbsp; by&nbsp; ER&nbsp; status&nbsp; (FGRR2,&nbsp; TNCR9), grade&nbsp; (FGRR2,&nbsp; TNCR9),&nbsp; bilateral&nbsp; dis- ease&nbsp; (2q35)&nbsp; and&nbsp; lobular&nbsp; versus&nbsp; ductal carcinoma&nbsp; (2q35)&nbsp; (Reeves&nbsp; et&nbsp; al.&nbsp; 2010). Genetically,&nbsp; sporadic&nbsp; breast&nbsp; cancer&nbsp; can be&nbsp; subdivided&nbsp; into&nbsp; 5&nbsp; distinct&nbsp; sub-groups and&nbsp; hormone&nbsp; receptor&nbsp; status&nbsp; plays&nbsp; a major&nbsp; role&nbsp; in&nbsp; discriminating&nbsp; these&nbsp; sub-groups&nbsp; (Sorlie&nbsp; et&nbsp; al.&nbsp; 2003).&nbsp; BRCA1-related&nbsp; tumours&nbsp; consistently&nbsp; group&nbsp; with sporadic&nbsp; tumours&nbsp; that&nbsp; are&nbsp; negative&nbsp; for ER,&nbsp; PR&nbsp; and&nbsp; HER2,&nbsp; &ldquo;triple&nbsp; negative&rdquo; (Sorlie&nbsp; et&nbsp; al.&nbsp; 2003).&nbsp; The&nbsp; most&nbsp; recent GWAS on&nbsp; BRCA1 carriers highlights the genetic&nbsp; heterogeneity&nbsp; of&nbsp; breast&nbsp; cancer, where&nbsp; only&nbsp; one&nbsp; locus&nbsp; was&nbsp; shown&nbsp; to&nbsp; in- fluence breast cancer risk and this locus had&nbsp; not&nbsp; previously&nbsp; been&nbsp; associated&nbsp; with sporadic&nbsp; disease.&nbsp; This&nbsp; locus&nbsp; was&nbsp; associated&nbsp; with&nbsp; the&nbsp; risk&nbsp; of&nbsp; developing&nbsp; ER- negative&nbsp; breast&nbsp; cancer&nbsp; and&nbsp; triple-negative&nbsp; breast&nbsp; cancer,&nbsp; but&nbsp; not&nbsp; ER-positive breast&nbsp; cancer&nbsp; (Antoniou&nbsp; et al.&nbsp; 2010). Other&nbsp; studies&nbsp; have&nbsp; also&nbsp; shown&nbsp; significantly&nbsp; stronger&nbsp; associations&nbsp; of&nbsp; many&nbsp; of the 13 identified variants with ER-positive disease&nbsp; compared&nbsp; to&nbsp; ER-negative&nbsp; disease&nbsp; and&nbsp; this&nbsp; is&nbsp; especially&nbsp; true&nbsp; of&nbsp; the polygenic&nbsp; model&nbsp; (Reeves&nbsp; et al. 2010). The&nbsp; most&nbsp; recent&nbsp; GWAS&nbsp; on ER-negative breast&nbsp; cancer&nbsp; found&nbsp; that&nbsp; there&nbsp; were&nbsp; no significant&nbsp; associations&nbsp; with&nbsp; any&nbsp; of&nbsp; the variants&nbsp; examined&nbsp; and found&nbsp; no evidence&nbsp; to&nbsp; suggest&nbsp; that&nbsp; ER-negative breast&nbsp; cancer&nbsp; has&nbsp; a polygenic&nbsp; basis&nbsp; for disease&nbsp; development,&nbsp; unlike&nbsp; ER-positive breast&nbsp; cancer&nbsp; (Li&nbsp; et al.&nbsp; 2010).&nbsp; Thus,&nbsp; recent GWAS on breast cancer have reiterated&nbsp; that&nbsp; importance&nbsp; of&nbsp; viewing&nbsp; breast cancer&nbsp; sub-types&nbsp; as&nbsp; distinct&nbsp; entities&nbsp; and underpin&nbsp; the genetically&nbsp; diverse&nbsp; pathways&nbsp; by&nbsp; which these tumour sub-groups develop.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	In&nbsp; conclusion,&nbsp; although&nbsp; the&nbsp; variants identified&nbsp; by&nbsp; GWAS&nbsp; have&nbsp; little&nbsp; clinical applicability&nbsp; at&nbsp; present,&nbsp; it&nbsp; is&nbsp; likely&nbsp; that more&nbsp; refined&nbsp; GWA&nbsp; studies&nbsp; of&nbsp; tumour sub-groups&nbsp; with&nbsp; defined&nbsp; pathological characteristics&nbsp; will&nbsp; shed&nbsp; more&nbsp; light&nbsp; on risk prediction in the future.</p>\r\n', 1);
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(460, 457, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	The&nbsp; CD151&nbsp; gene&nbsp; is&nbsp; a&nbsp; transmembrane molecule that has been characterized as a&nbsp; member&nbsp; of&nbsp; the&nbsp; tetraspanin&nbsp; family.&nbsp; The CD151&nbsp; transcript&nbsp; consists&nbsp; of&nbsp; 253&nbsp; amino acids that encode a 28 kDa protein. This gene&nbsp; is&nbsp; mapped&nbsp; to&nbsp; the&nbsp; human&nbsp; chromo- some 11p15.1. It is implicated in cell mo- tility,&nbsp; cell&nbsp; adhesion,&nbsp; and&nbsp; stability&nbsp; and&nbsp; for- mation of hemidesmosomes (Yauch et al. 1998).&nbsp; The&nbsp; role&nbsp; of&nbsp; CD151&nbsp; gene&nbsp; in&nbsp; cell motility&nbsp; highlighted&nbsp; its&nbsp; function&nbsp; as&nbsp; a&nbsp; pro- moter&nbsp; in&nbsp; tumour&nbsp; progression&nbsp; and&nbsp; metas- tasis&nbsp; events&nbsp; (Kohno&nbsp; et&nbsp; al.&nbsp; 2002).&nbsp; Over- expression&nbsp; of&nbsp; CD151&nbsp; was&nbsp; found&nbsp; to&nbsp; be associated&nbsp; with&nbsp; poor&nbsp; prognosis&nbsp; in&nbsp; non- small&nbsp; cell&nbsp; lung&nbsp; cancer,&nbsp; colon&nbsp; cancer&nbsp; and prostate&nbsp; cancer&nbsp; (Tokuhara&nbsp; et&nbsp; al.&nbsp; 2001; Hashida et al. 2003; Ang et al. 2004). In breast&nbsp; cancer,&nbsp; CD151&nbsp; is&nbsp; elevated&nbsp; in&nbsp; ap- proximately&nbsp; 31%&nbsp; of&nbsp; human&nbsp; breast&nbsp; can- cers&nbsp; and&nbsp; is&nbsp; significantly&nbsp; related&nbsp; to&nbsp; the high-grade&nbsp; (40%)&nbsp; and&nbsp; estrogen&nbsp; receptor negative (45%)&nbsp; subtypes&nbsp; (Yang&nbsp; et&nbsp; al. 2008). The CD151 gene is also a poten- tial&nbsp; marker&nbsp; in&nbsp; predicting&nbsp; histological grading&nbsp; in&nbsp; colon&nbsp; and&nbsp; prostate&nbsp; cancer (Hashida et al. 2003; Ang et al. 2004).&nbsp;</p>\r\n<p>\r\n	Study on the CD151 mRNA expression in&nbsp; correlation&nbsp; with&nbsp; clinicopathological features of breast cancer is rare. Herein, we investigated the expression of CD151 and&nbsp; correlated&nbsp; its&nbsp; expression&nbsp; with&nbsp; the histological&nbsp; features,&nbsp; ER&nbsp; status,&nbsp; PR&nbsp; sta- tus,&nbsp; c-erbB-2&nbsp; expression&nbsp; and tumour grades in breast cancer patients.</p>\r\n<p>\r\n	&nbsp;MATERIALS AND METHODS&nbsp; Patients and biopsy specimens&nbsp;&nbsp; A&nbsp; total&nbsp; of&nbsp; 45&nbsp; breast&nbsp; carcinomas&nbsp; were collected&nbsp; from&nbsp; fresh&nbsp; surgical&nbsp; resections from&nbsp; Kuala&nbsp; Lumpur&nbsp; Hospital,&nbsp; Universiti Kebangsaan&nbsp; Malaysia&nbsp; Medical&nbsp; Centre and&nbsp; Putrajaya&nbsp; Hospital.&nbsp; The&nbsp; biopsies were&nbsp; snap-frozen&nbsp; in&nbsp; liquid&nbsp; nitrogen&nbsp; prior to&nbsp; sample&nbsp; preparation.&nbsp; Tissues&nbsp; were touched&nbsp; on&nbsp; poly-L-Lysine&nbsp; coated&nbsp; slides and&nbsp; subjected&nbsp; for&nbsp; Diff&nbsp; Quick&nbsp; Staining (Imeb&nbsp; Inc.).&nbsp; Microscopic&nbsp; examination&nbsp; of touch&nbsp; preparations&nbsp; verified&nbsp; the&nbsp; presence of at least 70% of cancer cells in all sam- ples.&nbsp; Infiltrating&nbsp; ductal&nbsp; carcinoma&nbsp; (IDC) was&nbsp; diagnosed&nbsp; in&nbsp; 84.4%&nbsp; of&nbsp; the&nbsp; patients (N=39). Other tumours were identified as infiltrating&nbsp; lobular&nbsp; carcinoma&nbsp; (N=1),&nbsp; duc- tal&nbsp; carcinoma&nbsp; in-situ&nbsp; (DCIS)&nbsp; (N=2), mucinous&nbsp; carcinoma&nbsp; (N=1),&nbsp; mixed&nbsp; mu- cinous&nbsp; &amp;&nbsp; ductal&nbsp; carcinoma&nbsp; (N=1),&nbsp; and IDC&nbsp; with&nbsp; DCIS&nbsp; (N=1).&nbsp; Majority&nbsp; of&nbsp; the cases&nbsp; were&nbsp; from&nbsp; postmenopausal&nbsp; pa- tients. The mean and median ages of the patients are 50.8 and 52.0 years old, re- spectively.&nbsp; Tumour&nbsp; grade&nbsp; was&nbsp; evaluated using&nbsp; the&nbsp; Bloom-Richardson&nbsp; grading system.&nbsp; Nine,&nbsp; 24&nbsp; and&nbsp; 12&nbsp; tumours&nbsp; were classified&nbsp; as&nbsp; Grade&nbsp; 1,&nbsp; Grade&nbsp; 2&nbsp; and Grade&nbsp; 3&nbsp; tumours,&nbsp; respectively.&nbsp; The&nbsp; ER, PR and c-erbB-2 expression status for all samples&nbsp; were&nbsp; determined&nbsp; by&nbsp; immuno- histochemistry (IHC) and the results were as&nbsp; evaluated&nbsp; by&nbsp; the&nbsp; reporting&nbsp; patholo- gists.&nbsp; Tumours&nbsp; were&nbsp; regarded&nbsp; as&nbsp; ER&nbsp; or PR&nbsp; positive&nbsp; if&nbsp; &gt;10%&nbsp; of&nbsp; tumour&nbsp; cells&nbsp; ex- pressed&nbsp; nuclear&nbsp; positivity.&nbsp; On&nbsp; the&nbsp; other hand, strong membrane staining in &gt;10% cells&nbsp; was considered positive&nbsp; for&nbsp; c-erbB- 2. Breast carcinomas with known ER, PR and&nbsp; c-erbB-2&nbsp; expression&nbsp; were&nbsp; used&nbsp; as controls.&nbsp; The&nbsp; clones&nbsp; for&nbsp; ER,&nbsp; PR&nbsp; and&nbsp; c- erbB-2&nbsp; were&nbsp; 1DS&nbsp; (DAKO),&nbsp; PgR&nbsp; 636 (DAKO)&nbsp; and&nbsp; SP3&nbsp; (Neomarker),&nbsp; respec- tively.&nbsp; All&nbsp; IHC&nbsp; procedures&nbsp; were&nbsp; per- formed&nbsp; according&nbsp; to&nbsp; the&nbsp; manufacturer&rsquo;s recommendation.&nbsp; The&nbsp; clinical&nbsp; and&nbsp; histo- pathological&nbsp; data&nbsp; for&nbsp; the&nbsp; patients&nbsp; are listed in Table 1.&nbsp;</p>\r\n<p>\r\n	Ethics&nbsp; approval&nbsp; was&nbsp; obtained&nbsp; from Medical&nbsp; Research&nbsp; &amp;&nbsp; Ethics&nbsp; Committee, Ministry&nbsp; of&nbsp; Health&nbsp; Malaysia&nbsp; to&nbsp; perform this study.&nbsp; RNA isolation&nbsp;&nbsp; Minced&nbsp; tissues&nbsp; were&nbsp; placed&nbsp; directly&nbsp; in Trizol&nbsp; Reagent&nbsp; (Invitrogen),&nbsp; homoge- nized,&nbsp; and total RNAs&nbsp; were isolated and purified&nbsp; through&nbsp; RNeasy&nbsp; columns&nbsp; (QIA- GEN)&nbsp; according&nbsp; to&nbsp; the&nbsp; manufacturer&rsquo;s recommendation. The integrity of the pu- rified total RNA was assessed by visuali- zation&nbsp; of&nbsp; the&nbsp; 28S/18S&nbsp; ribosomal&nbsp; RNA ratio&nbsp; on&nbsp; 1%&nbsp; agarose&nbsp; denaturing&nbsp; gel&nbsp; and the quantity was assessed based on ab- sorbance at 260nm and 280nm in Nano- drop.&nbsp;&nbsp; cDNA synthesis&nbsp; Total RNA was converted to cDNA using AMV&nbsp; reverse&nbsp; transcriptase&nbsp; First&nbsp; strand cDNA&nbsp; synthesis&nbsp; kit&nbsp; for&nbsp; RT-PCR&nbsp; (AMV), Roche Diagnostics]. The cDNA synthesis reactions&nbsp; were&nbsp; performed&nbsp; in&nbsp; 20&micro;L&nbsp; vo- lumes&nbsp; containing&nbsp; 200ng&nbsp; of&nbsp; total&nbsp; RNAs, 1X&nbsp; reaction&nbsp; buffer,&nbsp; 5mM&nbsp; MgCl2,&nbsp; 1mM dNTPs,&nbsp; 3.2&micro;g&nbsp; of&nbsp; random&nbsp; primer&nbsp; p(dN)6 , 50 units RNase inhibitor and &ge; 20 units of AMV&nbsp; reverse&nbsp; transcriptase.&nbsp; Total&nbsp; RNAs with&nbsp; the&nbsp; appropriate&nbsp; volume&nbsp; of&nbsp; nuclease free&nbsp; water&nbsp; were&nbsp; first&nbsp; incubated&nbsp; at&nbsp; 65&deg;C for&nbsp; 15min.&nbsp; The&nbsp; master&nbsp; mix&nbsp; was&nbsp; then added to the tube and incubated at 25&deg;C for&nbsp; 10min,&nbsp; and&nbsp; then&nbsp; at&nbsp; 42&deg;C&nbsp; for&nbsp; 1h.&nbsp; The reaction&nbsp; was&nbsp; then&nbsp; incubated&nbsp; at&nbsp; 99&deg;C&nbsp; for 5min&nbsp; to&nbsp; denature&nbsp; the&nbsp; enzymes.&nbsp; Finally, the reaction was cooled to 4&deg;C for 5min.&nbsp;</p>\r\n<p>\r\n	Polymerase chain reaction (PCR)&nbsp;&nbsp;</p>\r\n<p>\r\n	Polymerase&nbsp; chain&nbsp; reaction&nbsp; was&nbsp; per- formed&nbsp; to&nbsp; amplify&nbsp; GAPDH&nbsp; and&nbsp; CD151 genes. The PCR reaction was carried out in&nbsp; 25&micro;L&nbsp; volumes&nbsp; containing&nbsp; 4-6&micro;g&nbsp; of cDNA&nbsp; generated&nbsp; from&nbsp; breast&nbsp; tumour, 0.4pmole/&micro;L&nbsp; of&nbsp; primers&nbsp; and&nbsp; 1X&nbsp; PCR master&nbsp; mix&nbsp; (Panomics).&nbsp; The&nbsp; reaction mixture&nbsp; was&nbsp; pre-incubated&nbsp; at&nbsp; 95&deg;C&nbsp; for&nbsp; 5 min,&nbsp; followed&nbsp; by&nbsp; 40-45&nbsp; cycles&nbsp; of&nbsp; amplifi- cation at 94&deg;C for 30s, 54&deg;C for 30s and 72&deg;C&nbsp; for&nbsp; 30s.&nbsp; A&nbsp; negative&nbsp; control&nbsp; was&nbsp; in- cluded in each PCR to exclude contami- nation.&nbsp; PCR&nbsp; products&nbsp; for&nbsp; GAPDH&nbsp; and CD151 genes were purified (Qiagen PCR purification&nbsp; kit).&nbsp; Then,&nbsp; 10X&nbsp; serial&nbsp; dilution were prepared for GAPDH and CD151 to generate standard curves in qRT-PCR.&nbsp;</p>\r\n<p>\r\n	Real-time RT-PCR (qRT-PCR)&nbsp;</p>\r\n<p>\r\n	Real-time&nbsp; RT-PCR&nbsp; was&nbsp; performed&nbsp; using LightCycler&reg;&nbsp; LC480&nbsp; Probe&nbsp; Master (Roche&nbsp; Diagnostics).&nbsp; Probes&nbsp; for&nbsp; CD151 (cat.&nbsp; no.:&nbsp; 04688660001)&nbsp; and&nbsp; house- keeping&nbsp; gene,&nbsp; GAPDH&nbsp; (cat.&nbsp; no.: 04688589001) were purchased from Uni- versal&nbsp; ProbeLibrary&nbsp; (Roche&nbsp; Diagnostics) and&nbsp; the&nbsp; primers&nbsp; were&nbsp; synthesized&nbsp; from Bioneer.&nbsp; The&nbsp; primer&nbsp; sequences&nbsp; were CD151-F:&nbsp; 5&rsquo;-CTG&nbsp; CGC&nbsp; CTG&nbsp; TAC&nbsp; TTC ATC&nbsp; G-3&rsquo;&nbsp; and&nbsp; CD151-R:&nbsp; 5&rsquo;-TTC&nbsp; TCC TTG&nbsp; AGC&nbsp; TCC&nbsp; GTG&nbsp; TT-3&rsquo;;&nbsp; GAPDH-F: 5&rsquo;-CTC&nbsp; TGC&nbsp; TCC TCC&nbsp; TGT TCG&nbsp; AC-3&rsquo; and&nbsp; GAPDH-R:&nbsp; 5&rsquo;-ACG&nbsp; ACC&nbsp; AAA&nbsp; TCC GTT&nbsp; GAC&nbsp; TC-3&rsquo;.&nbsp; qRT-PCR&nbsp; was&nbsp; carried out&nbsp; in 20&micro;L&nbsp; volumes&nbsp; containing&nbsp; 4&micro;g&nbsp; of cDNA,&nbsp; 1X&nbsp; LightCycler&reg;&nbsp; LC480&nbsp; Probe Master,&nbsp; 2&micro;L&nbsp; of&nbsp; probes&nbsp; and&nbsp; 0.8&ndash;1.0pmol of each primer. qRT-PCR was carried out on&nbsp; LightCycler&reg; &nbsp; LC480&nbsp; using&nbsp; a&nbsp; 96&nbsp; well plate&nbsp; (Roche&nbsp; Diagnostics).&nbsp; All&nbsp; reactions were&nbsp; run&nbsp; in&nbsp; duplicates.&nbsp; The&nbsp; amplification program&nbsp; consisted&nbsp; of&nbsp; pre-incubation&nbsp; at 95&deg;C&nbsp; with&nbsp; a&nbsp; 10min&nbsp; hold,&nbsp; denaturation&nbsp; at 95&deg;C&nbsp; with&nbsp; a&nbsp; 10s&nbsp; hold,&nbsp; followed&nbsp; by&nbsp; an- nealing&nbsp; at&nbsp; 54&deg;C&nbsp; with&nbsp; a&nbsp; 10s&nbsp; hold&nbsp; (40 cycles)&nbsp; and&nbsp; extension&nbsp; at&nbsp; 72&deg;C&nbsp; with&nbsp; a&nbsp; 1s hold.&nbsp; The&nbsp; sizes&nbsp; of&nbsp; the&nbsp; amplicons&nbsp; were 107&nbsp; bp&nbsp; and&nbsp; 112&nbsp; bp&nbsp; for&nbsp; CD151&nbsp; and GAPDH, respectively.&nbsp;&nbsp;</p>\r\n<p>\r\n	Statistical analysis&nbsp;</p>\r\n<p>\r\n	Standard curves for GAPDH and CD151 were&nbsp; optimized&nbsp; by&nbsp; using&nbsp; diluted&nbsp; PCR products.&nbsp; Gene&nbsp; expressions&nbsp; of&nbsp; each sample&nbsp; were&nbsp; obtained&nbsp; by&nbsp; comparing&nbsp; the crossing&nbsp; points&nbsp; (Ct)&nbsp; with&nbsp; the&nbsp; standard curve.&nbsp; The&nbsp; obtained&nbsp; gene&nbsp; expression&nbsp; or concentration&nbsp; of&nbsp; CD151&nbsp; gene&nbsp; was&nbsp; then normalized&nbsp; with&nbsp; GAPDH.&nbsp; Mean&nbsp; expres- sions&nbsp; of&nbsp; CD151&nbsp; for&nbsp; each&nbsp; case&nbsp; were grouped&nbsp; according&nbsp; to&nbsp; the&nbsp; ER&nbsp; status,&nbsp; PR status,&nbsp; c-erbB-2&nbsp; expression&nbsp; and&nbsp; tumour grades.&nbsp; The&nbsp; significant&nbsp; association&nbsp; of CD151&nbsp; expression&nbsp; with&nbsp; the&nbsp; ER&nbsp; status, PR status and c-erbB-2 expression were determined&nbsp; by&nbsp; t-test&nbsp; unequal&nbsp; variances while&nbsp; the&nbsp; relation&nbsp; of&nbsp; CD151&nbsp; and&nbsp; tumour status was determined by ANOVA.</p>\r\n<p>\r\n	<strong>RESULTS </strong></p>\r\n<p>\r\n	&nbsp;In our study, PCR efficiencies for CD151 and&nbsp; GAPDH&nbsp; were&nbsp; 1.674&nbsp; and&nbsp; 1.758,&nbsp; re- spectively.&nbsp; The&nbsp; normalized&nbsp; mRNA&nbsp; ex- pressions&nbsp; of&nbsp; CD151&nbsp; for&nbsp; all&nbsp; the&nbsp; samples examined in this study are listed in Table 1. The mean CD151 expression in differ- ent&nbsp; histological&nbsp; groups&nbsp; are&nbsp; illustrated&nbsp; in Figure 1.</p>\r\n<p>\r\n	Our&nbsp; preliminary&nbsp; real&nbsp; time&nbsp; RT-PCR&nbsp; re- sults&nbsp; revealed&nbsp; that&nbsp; there&nbsp; was&nbsp; significant difference&nbsp; between&nbsp; CD151&nbsp; expression and&nbsp; the&nbsp; ER&nbsp; status&nbsp; (p=0.012)&nbsp; and&nbsp; PR status (p=0.009) of breast tumour. Higher CD151&nbsp; mRNA&nbsp; level&nbsp; was&nbsp; detected&nbsp; in&nbsp; ER positive and PR positive breast tumours. Conversely,&nbsp; CD151&nbsp; expression&nbsp; was&nbsp; not associated&nbsp; with&nbsp; the&nbsp; tumour&nbsp; grade (p=0.057)&nbsp; and&nbsp; c-erbB-2&nbsp; expression (p=0.060) in our present study.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	&nbsp;Several&nbsp; members&nbsp; of&nbsp; the&nbsp; tetraspanin&nbsp; su- perfamily&nbsp; have&nbsp; been&nbsp; identified&nbsp; as&nbsp; me- tastasis&nbsp; suppressor&nbsp; genes&nbsp; in&nbsp; cancer pathways.&nbsp; The&nbsp; CD151&nbsp; gene&nbsp; was&nbsp; re- ported&nbsp; to&nbsp; be&nbsp; the&nbsp; first&nbsp; member&nbsp; of&nbsp; the&nbsp; te- traspanin superfamily, which plays a cru-cial&nbsp; role&nbsp; as&nbsp; promoter&nbsp; in&nbsp; cancer&nbsp; metasta-sis&nbsp; (Testa&nbsp; et&nbsp; al.&nbsp; 1999).&nbsp; Notably,&nbsp; CD151 gene&nbsp; mediates&nbsp; cell&nbsp; migration&nbsp; and&nbsp; facili-tates invasion by regulating laminin (LN)- binding&nbsp; intergrins&nbsp; such&nbsp; as&nbsp; &alpha;3&beta;1,&nbsp; &alpha;6&beta;1, &alpha;6&beta;4,&nbsp; and&nbsp; &alpha;7&beta;1&nbsp; through&nbsp; formation&nbsp; of CD151-integrin&nbsp; complexes&nbsp; called&nbsp; tetras- panin-enriched microdomains (TEM) (Liu et al. 2007). Recent study on knockdown CD151&nbsp; expression&nbsp; in&nbsp; breast&nbsp; cancer&nbsp; cell showed&nbsp; that&nbsp; hepatocyte&nbsp; growth&nbsp; factor (HGF)&nbsp; receptor/c-met&nbsp; signaling&nbsp; pathway might&nbsp; be&nbsp; changed&nbsp; by&nbsp; the&nbsp; decreased&nbsp; Akt phosphorylation&nbsp; in&nbsp; cells&nbsp; lacking&nbsp; CD151 (Klosek&nbsp; et&nbsp; al.&nbsp; 2009).&nbsp; C-met&nbsp; signaling pathway plays a crucial role in mitogenic, proliferative,&nbsp; morphogenic&nbsp; and&nbsp; angi- ogenic activities. It correlates with breast cancer progression and metastasis (Park et al. 2005). The ability of CD151 in me- diating&nbsp; cell&nbsp; migration&nbsp; suggested&nbsp; that CD151&nbsp; is&nbsp; possibly&nbsp; involved&nbsp; in&nbsp; a&nbsp; molecu-lar&nbsp; mechanism&nbsp; that&nbsp; could&nbsp; lead&nbsp; to&nbsp; metas-tatic&nbsp; progression&nbsp; of&nbsp; cancerous&nbsp; cells&nbsp; (Ha-shida&nbsp; et&nbsp; al.&nbsp; 2003).&nbsp; This&nbsp; hypothesis&nbsp; is proven&nbsp; by&nbsp; Novitskaya&nbsp; et&nbsp; al.&nbsp; (2010)&nbsp; and Sadej&nbsp; et&nbsp; al.&nbsp; (2010).&nbsp; They&nbsp; reported&nbsp; that CD151&nbsp; expression&nbsp; is&nbsp; increased&nbsp; in&nbsp; inva-sive&nbsp; breast&nbsp; carcinomas&nbsp; and&nbsp; higher&nbsp; tu-mour&nbsp; grade&nbsp; and&nbsp; node&nbsp; metastasis.&nbsp; Addi- tionally,&nbsp; CD151&nbsp; is&nbsp; confirmed&nbsp; as&nbsp; a&nbsp;&nbsp; prognostic marker of outcome in lung and prostate&nbsp; cancers&nbsp; (Tokuhara&nbsp; et&nbsp; al.&nbsp; 2001; Ang et al. 2004).&nbsp;</p>\r\n<p>\r\n	Although&nbsp; previous&nbsp; studies&nbsp; have&nbsp; con- firmed&nbsp; the&nbsp; ability&nbsp; of&nbsp; CD151&nbsp; in&nbsp; facilitating cell migration and spreading, very limited information&nbsp; is&nbsp; available&nbsp; on&nbsp; the&nbsp; relation- ship&nbsp; between&nbsp; CD151&nbsp; expression&nbsp; and hormone&nbsp; profile&nbsp; such&nbsp; as&nbsp; estrogen&nbsp; re- ceptor&nbsp; and&nbsp; progesterone&nbsp; receptor&nbsp; status in&nbsp; breast&nbsp; cancer&nbsp; patients.&nbsp; The&nbsp; present study&nbsp; revealed&nbsp; that&nbsp; there&nbsp; was&nbsp; significant association&nbsp; between&nbsp; CD151&nbsp; expression and ER positive (p&lt;0.05) and PR positive subtypes&nbsp; (p&lt;0.01).&nbsp; CD151&nbsp; expression was&nbsp; higher&nbsp; in&nbsp; ER&nbsp; positive&nbsp; compared&nbsp; to ER negative breast tumours. It is also ex- pressed&nbsp; at&nbsp; a&nbsp; higher&nbsp; level&nbsp; in&nbsp; PR&nbsp; positive relative&nbsp; to&nbsp; the&nbsp; PR&nbsp; negative&nbsp; breast&nbsp; tu- mours.&nbsp; Since&nbsp; ER&nbsp; positive&nbsp; breast&nbsp; cancer patients&nbsp; have&nbsp; better&nbsp; survival&nbsp; than&nbsp; ER negative&nbsp; patients,&nbsp; we&nbsp; suggest&nbsp; that&nbsp; the expression&nbsp; of&nbsp; CD151&nbsp; could&nbsp; be&nbsp; another predictor&nbsp; for&nbsp; a&nbsp; better&nbsp; survival&nbsp; in&nbsp; breast cancer&nbsp; patients.&nbsp; Our&nbsp; findings&nbsp; is&nbsp; contrary with previous findings by Tokuhara et al. (2001),&nbsp; Ang&nbsp; et&nbsp; al.&nbsp; (2004)&nbsp; and&nbsp; Zijlstra&nbsp; et al. (2008) which said that CD151 expres- sion&nbsp; is&nbsp; increased&nbsp; in&nbsp; advanced&nbsp; stage&nbsp; of the&nbsp; disease&nbsp; and&nbsp; shorter&nbsp; overall&nbsp; survival. However, our result is consistent with the findings&nbsp; reported&nbsp; by&nbsp; Voss&nbsp; et&nbsp; al.&nbsp; (2011). Voss et al. (2011) postulated that CD151 expression&nbsp; may&nbsp; prevent&nbsp; the&nbsp; transition&nbsp; of endometrial cancer to a more aggressive phenotype.&nbsp; They&nbsp; found&nbsp; that&nbsp; high&nbsp; CD151 expression&nbsp; is&nbsp; correlated&nbsp; with&nbsp; improved survival in the patients. How CD151 pre- vent&nbsp; the tumour&nbsp; cell&nbsp; transition&nbsp; is&nbsp; un- known.&nbsp; The&nbsp; pathway&nbsp; or&nbsp; biochemical function of CD151 in estrogen dependent breast&nbsp; cancer&nbsp; is&nbsp; still&nbsp; unknown&nbsp; and&nbsp; re- mains to be investigated.&nbsp;</p>\r\n<p>\r\n	Yang&nbsp; et al. (2008)&nbsp; had&nbsp; suggested possible connection between high CD151 transcript&nbsp; level&nbsp; and c-erbB-2&nbsp; expression in breast tumour (Yang&nbsp; et al. 2008). Our study revealed that there is no significant difference&nbsp; between&nbsp; CD151&nbsp; expression and&nbsp; c-erbB-2&nbsp; expression.&nbsp; Nevertheless, our&nbsp; findings&nbsp; revealed&nbsp; lower&nbsp; CD151 mRNA&nbsp; level in triple&nbsp; negative&nbsp; (ER,&nbsp; PR and&nbsp; c-erbB-2&nbsp; negative)&nbsp; breast&nbsp; carcino- mas.&nbsp; The&nbsp; average&nbsp; CD151&nbsp; expression level&nbsp; in triple&nbsp; negative&nbsp; breast&nbsp; tumours (113,&nbsp; 147,&nbsp; 164, 168 and&nbsp; 173)&nbsp; is&nbsp; 0.14, which&nbsp; is&nbsp; approximately&nbsp; one&nbsp; fold&nbsp; lower that&nbsp; other&nbsp; types&nbsp; of&nbsp; tumours&nbsp; (0.27).&nbsp; This observation&nbsp; suggests&nbsp; the important&nbsp; role of&nbsp; CD151&nbsp; in&nbsp; the&nbsp; pathogenesis&nbsp; of&nbsp; triple negative&nbsp; breast&nbsp; tumours.&nbsp; Thus,&nbsp; c-erbB-2 expression&nbsp; may&nbsp; not&nbsp; link&nbsp; directly&nbsp; with CD151 expression but its expression de- pends on ER and PR status as well.&nbsp;</p>\r\n<p>\r\n	Several&nbsp; research&nbsp; teams&nbsp; have&nbsp; found that elevated CD151 mRNA level is asso- ciated&nbsp; with&nbsp; a&nbsp; more&nbsp; advanced&nbsp; and&nbsp; ag- gressive stage of the disease. In prostate cancer,&nbsp; CD151&nbsp; expression&nbsp; was&nbsp; signifi- cantly&nbsp; different&nbsp; among&nbsp; well,&nbsp; moderately and&nbsp; poorly&nbsp; differentiated&nbsp; tumours&nbsp; (Ha- shida et al. 2003; Ang et al. 2004). How- ever, CD151 expression is not correlated with&nbsp; breast&nbsp; tumour&nbsp; grades&nbsp; in&nbsp; our&nbsp; study (p&lt;0.05).&nbsp; Our&nbsp; result&nbsp; is&nbsp; unexpected.&nbsp; The reason for inconsistency of our result with previous&nbsp; findings&nbsp; could&nbsp; be&nbsp; due&nbsp; to&nbsp; the sample size in our study. More than 70% of&nbsp; the&nbsp; subjects&nbsp; used&nbsp; in&nbsp; this&nbsp; study&nbsp; were grade 1 and 2 breast tumours (N=33/45). Our&nbsp; results&nbsp; showed&nbsp; that&nbsp; elevated CD151&nbsp; expression&nbsp; may&nbsp; be&nbsp; associated be&nbsp; another&nbsp; important&nbsp; prognostic&nbsp; marker and&nbsp; treatment&nbsp; decision&nbsp; in&nbsp; estrogen&nbsp; de- pendent&nbsp; breast&nbsp; cancer&nbsp; patients.&nbsp; Exten- sive&nbsp; study&nbsp; of&nbsp; CD151&nbsp; mRNA&nbsp; level&nbsp; in es- trogen-depe ndent&nbsp; breast&nbsp; tumours&nbsp; is&nbsp; cru- cial&nbsp; to&nbsp; delineate&nbsp; its&nbsp; role&nbsp; in&nbsp; breast&nbsp; cancer pathogenesis.&nbsp;</p>\r\n<p>\r\n	Our&nbsp; study&nbsp; however&nbsp; has&nbsp; limitations&nbsp; due to the inter-observer variability of the pa- thologists in the histopathology examina- tion&nbsp; of&nbsp; ER,&nbsp; PR,&nbsp; c-erbB-2&nbsp; and&nbsp; tumour grade.&nbsp; Therefore,&nbsp; larger&nbsp; sample&nbsp; size&nbsp; is needed&nbsp; to&nbsp; verify&nbsp; the&nbsp; correlation&nbsp; between CD151 expression level and ER and PR status.&nbsp; Alternatively,&nbsp; the&nbsp; role&nbsp; of&nbsp; CD151 gene&nbsp; could&nbsp; be&nbsp; determined&nbsp; by&nbsp; studying the function of the gene in breast cancer cell line.</p>\r\n<p>\r\n	<strong>&nbsp;ACKNOWLEDGEMENTS </strong></p>\r\n<p>\r\n	&nbsp;This&nbsp; research&nbsp; was&nbsp; supported&nbsp; by&nbsp; a&nbsp; grant from the Ministry of Science and Innova-tion&nbsp; (MOSTI).&nbsp; The&nbsp; authors&nbsp; would&nbsp; like&nbsp; to thank&nbsp; the&nbsp; Director&nbsp; General&nbsp; of&nbsp; Health Malaysia for his permission to publish the study&nbsp; findings. We&nbsp; also&nbsp; like&nbsp; to&nbsp; thank&nbsp; the Director&nbsp; of&nbsp; the&nbsp; Institute&nbsp; for&nbsp; Medical&nbsp; Re-search for her support in the approval of publishing the research findings.</p>\r\n', 1),
(461, 458, '<p>\r\n	<strong>INTRODUCTION&nbsp; </strong></p>\r\n<p>\r\n	Prior&nbsp; to&nbsp; this&nbsp; study,&nbsp; information&nbsp; about&nbsp; or- thodontic&nbsp; treatment&nbsp; was&nbsp; usually&nbsp; de- scribed&nbsp; verbally&nbsp; to&nbsp; patients&nbsp; during screening&nbsp; and&nbsp; treatment&nbsp; in&nbsp; the&nbsp; UKM Dental&nbsp; Clinic.&nbsp; The&nbsp; purpose&nbsp; of&nbsp; the screening&nbsp; was&nbsp; to&nbsp; identify&nbsp; patients&nbsp; who were&nbsp; suitable&nbsp; to&nbsp; undergo&nbsp; orthodontic treatment.&nbsp; Initial&nbsp; consultation&nbsp; was&nbsp; also carried&nbsp; out&nbsp; to&nbsp; facilitate&nbsp; the&nbsp; patients&nbsp; in making informed decisions.&nbsp;</p>\r\n<p>\r\n	The&nbsp; patients&nbsp; who&nbsp; attended&nbsp; the&nbsp; screening were commonly teenagers and young adults&nbsp; of&nbsp; various&nbsp; ethnicity.&nbsp; Sometimes, adult patients attended the screening be- cause&nbsp; they&nbsp; require&nbsp; adjunctive&nbsp; treatment for&nbsp; periodontal&nbsp; disease&nbsp; or&nbsp; for&nbsp; restorative purposes.&nbsp;</p>\r\n<p>\r\n	Orthodontic&nbsp; screening&nbsp; was&nbsp; carried&nbsp; out by&nbsp; fifth&nbsp; year&nbsp; dental&nbsp; students,&nbsp; who&nbsp; were taught&nbsp; to&nbsp; convey&nbsp; important&nbsp; information regarding&nbsp; orthodontic&nbsp; treatment&nbsp; to&nbsp; the patients.&nbsp; Nevertheless,&nbsp; there&nbsp; was&nbsp; no clear&nbsp; guideline&nbsp; on&nbsp; what&nbsp; and&nbsp; how&nbsp; messages should be conveyed to the patient. Hence,&nbsp; the&nbsp; amount&nbsp; of&nbsp; information&nbsp; given was&nbsp; dependent&nbsp; on&nbsp; the&nbsp; individual&nbsp; clinician. This contributed to varying levels of retention among patients.</p>\r\n<p>\r\n	Therefore,&nbsp; concern&nbsp; was&nbsp; raised&nbsp; regard- ing&nbsp; the&nbsp; relationship&nbsp; of&nbsp; the&nbsp; mode&nbsp; of&nbsp; deli- very to the retention of information about orthodontic&nbsp; treatment.&nbsp; Since&nbsp; it was&nbsp; im- portant&nbsp; for&nbsp; patients&nbsp; to&nbsp; understand&nbsp; the various&nbsp; aspects&nbsp; of&nbsp; orthodontic&nbsp; treatment before&nbsp; they&nbsp; make&nbsp; their&nbsp; commitment&nbsp; to- wards&nbsp; it, it was&nbsp; crucial&nbsp; to&nbsp; evaluate&nbsp; the methods&nbsp; of&nbsp; communication&nbsp; which&nbsp; aid&nbsp; in the&nbsp; patients&rsquo;&nbsp; ability&nbsp; to&nbsp; recall&nbsp; essential&nbsp; in- formation about orthodontic treatment.</p>\r\n<p>\r\n	The&nbsp; aim&nbsp; of&nbsp; the&nbsp; study&nbsp; was&nbsp; to&nbsp; compare patients&rsquo;&nbsp; level&nbsp; of&nbsp; recall&nbsp; on&nbsp; verbal&nbsp; and written&nbsp; information.&nbsp; To&nbsp; achieve&nbsp; that,&nbsp; it was&nbsp; necessary&nbsp; to&nbsp; validate&nbsp; translation&nbsp; of the&nbsp; information&nbsp; leaflet,&nbsp; develop&nbsp; and&nbsp; test the questionnaire, as well as evaluate the patients&rsquo; ability to recall the information.</p>\r\n<p>\r\n	Locker&nbsp; (1989)&nbsp; stated&nbsp; effective&nbsp; commu- nication&nbsp; facilitates&nbsp; diagnosis&nbsp; making&nbsp; and treatment&nbsp; planning,&nbsp; and&nbsp; has&nbsp; a&nbsp; huge&nbsp; influence&nbsp; on&nbsp; the&nbsp; result&nbsp; of&nbsp; the&nbsp; treatment. Greater&nbsp; patient&nbsp; satisfaction&nbsp; with&nbsp; less&nbsp; patient&nbsp; litigation&nbsp; was&nbsp; reported.&nbsp; Sufficient understanding&nbsp; of&nbsp; the&nbsp; treatment&nbsp; process eased&nbsp; decision&nbsp; making&nbsp; and&nbsp; consent (Cannavina&nbsp; et&nbsp; al.&nbsp; 2000).&nbsp; Furthermore, effective&nbsp; communication&nbsp; and&nbsp; good&nbsp; understanding of the information given were important to achieve compliance and cooperation&nbsp; (Anderson&nbsp; &amp; Freer&nbsp; 2005). Whether&nbsp; or&nbsp; not&nbsp; a patient&nbsp; was&nbsp; satisfied with&nbsp; the&nbsp; clinician&nbsp; was&nbsp; influenced&nbsp; by&nbsp; the information&nbsp; and consultation&nbsp; given&nbsp; and understood (Ley 1988).</p>\r\n<p>\r\n	In particular, patients and parents some- times&nbsp; faced&nbsp; difficulty&nbsp; in fully&nbsp; realizing the consequences and requirements of orthodontic&nbsp; treatment&nbsp; (Pratelli&nbsp; et&nbsp; al. 1996). Insufficient&nbsp; information&nbsp; about orthodontic treatment and lack of communication can cause&nbsp; lack&nbsp; of&nbsp; patient&nbsp; cooperation&nbsp; and premature&nbsp; termination&nbsp; of&nbsp; orthodontic treatment (Brattstrom et al. 1991).&nbsp;</p>\r\n<p>\r\n	Unfortunately,&nbsp; the amount&nbsp; of&nbsp; informa- tion received by the patients is generally perceived&nbsp; unsatisfactory&nbsp; (Newton&nbsp; 1995), even&nbsp; though&nbsp; information&nbsp; giving&nbsp; is&nbsp; noted as an important communication skill rele- vant&nbsp; to dentists.&nbsp; Successful&nbsp; communica- tion often involved the patient&rsquo;s exposure to&nbsp; the messages,&nbsp; drawing&nbsp; attention&nbsp; to personal&nbsp; benefits,&nbsp; understanding&nbsp; the messages,&nbsp; accepting&nbsp; and&nbsp; retaining&nbsp; the messages (Ashford &amp; Blinkhorn 1999).&nbsp;</p>\r\n<p>\r\n	The&nbsp; most&nbsp; common&nbsp; form&nbsp; of&nbsp; imparting dental&nbsp; knowledge&nbsp; is&nbsp; by&nbsp; verbal&nbsp; provision of&nbsp; information.&nbsp; Nevertheless,&nbsp; physical barriers&nbsp; and&nbsp; noise&nbsp; of&nbsp; handpieces&nbsp; in&nbsp; the clinic hinder effective verbal communication (Humphris &amp; Ling 2000).</p>\r\n<p>\r\n	Additionally,&nbsp; in&nbsp; a&nbsp; multicultural&nbsp; society, communication&nbsp; difficulties&nbsp; frequently&nbsp; exist when the patients have limited understanding&nbsp; of&nbsp; the&nbsp; language&nbsp; use&nbsp; by&nbsp; the dentists&nbsp; (Williams&nbsp; et&nbsp; al.&nbsp; 1995).&nbsp; To&nbsp; over- come&nbsp; the&nbsp; language&nbsp; barrier,&nbsp; the&nbsp; use&nbsp; of interpreters is recommended. Non-verbal adjuncts&nbsp; such&nbsp; as&nbsp; dental&nbsp; health&nbsp; publica- tions&nbsp; may&nbsp; be&nbsp; used&nbsp; to&nbsp; enhance&nbsp; information&nbsp; communication,&nbsp; provided&nbsp; the&nbsp; pa- tients can read (Goldsmith et al. 2005).</p>\r\n<p>\r\n	A German study (Chatziandroni-Frey et al.&nbsp; 2000)&nbsp; found&nbsp; that&nbsp; briefing&nbsp; media&nbsp; such as&nbsp; demonstration&nbsp; models&nbsp; and&nbsp; leaflets were&nbsp; used&nbsp; primarily&nbsp; in the&nbsp; orthodontists&rsquo; waiting&nbsp; room&nbsp; and surgery.&nbsp; Space&nbsp; taking<br />\r\n	media&nbsp; (video-films,&nbsp; computers)&nbsp; and books were rarely used. The advantages of&nbsp; information&nbsp; leaflets&nbsp; are&nbsp; non-intrusive, inexpensive&nbsp; and&nbsp; time&nbsp; neutral&nbsp; (Humphris &amp; Field 2003). However, its effectiveness is&nbsp; dependent&nbsp; on&nbsp; its&nbsp; readability&nbsp; (Albert&nbsp; &amp; Chadwick 1992; Roger 2000).&nbsp;</p>\r\n<p>\r\n	Also,&nbsp; a study&nbsp; found&nbsp; that&nbsp; patients&nbsp; who were&nbsp; motivated&nbsp; to read&nbsp; the leaflets showed significant improvement in know- ledge&nbsp; when&nbsp; compared&nbsp; to&nbsp; patients&nbsp; who were passively given the leaflets (O&rsquo;Neill et al.&nbsp; 1996). Hence,&nbsp; it appeared&nbsp; that the provision&nbsp; of&nbsp; a leaflet&nbsp; alone&nbsp; gave&nbsp; limited benefits. Often it must be combined&nbsp; with verbal information.&nbsp;</p>\r\n<p>\r\n	Humphris&nbsp; &amp; Ling (2000)&nbsp; suggested factors such as patient education level is not&nbsp; strongly&nbsp; related&nbsp; to the&nbsp; degree&nbsp; of&nbsp; re- call. However, studies focusing on reten- tion of orthodontic information had shown varying&nbsp; results.&nbsp; Parents&rsquo;&nbsp; educational level was marginally associated with their recall&nbsp; of&nbsp; risks,&nbsp; but their&nbsp; vocabulary&nbsp; level was&nbsp; significantly&nbsp; correlated&nbsp; with&nbsp; the number&nbsp; of&nbsp; reasons&nbsp; for&nbsp; treatment&nbsp; that they would recall (Baird &amp; Kiyak 2003).</p>\r\n<p>\r\n	In&nbsp; one study,&nbsp; written,&nbsp; verbal&nbsp; and visual methods&nbsp; were&nbsp; tested&nbsp; but&nbsp; little&nbsp; difference was&nbsp; found&nbsp; (Thomson&nbsp; et al.&nbsp; 2001).&nbsp; Nevertheless,&nbsp; the&nbsp; study&nbsp; suggested&nbsp; verbal information&nbsp; should&nbsp; always&nbsp; be supple- mented by written and/ or visual information.&nbsp;</p>\r\n<p>\r\n	In contrast, another study found that the participants&nbsp; who&nbsp; were&nbsp; given&nbsp; information leaflets&nbsp; about&nbsp; orthodontic&nbsp; treatment&nbsp; per- formed&nbsp; poorer&nbsp; in&nbsp; recalling&nbsp; the informa- tion, when compared to participants who were&nbsp; given&nbsp; mind&nbsp; maps&nbsp; or&nbsp; acronyms (Newton &amp; Thickett 2006).&nbsp;&nbsp;</p>\r\n<p>\r\n	Similarly,&nbsp; when&nbsp; a&nbsp; group&nbsp; of&nbsp; patients&nbsp; receiving computer-based visual information was compared to another group receiving information&nbsp; leaflets,&nbsp; the&nbsp; computer-based visual&nbsp; information&nbsp; was&nbsp; superior&nbsp; to&nbsp; information leaflets (Patel et al 2008).&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS&nbsp; </strong></p>\r\n<p>\r\n	A&nbsp; questionnaire&nbsp; based&nbsp; study&nbsp; was&nbsp; carried out&nbsp; at&nbsp; the&nbsp; Faculty&nbsp; of&nbsp; Dentistry,&nbsp; Universiti Kebangsaan&nbsp; Malaysia&nbsp; (UKM).&nbsp; Ethics&nbsp; ap- proval&nbsp; was&nbsp; obtained&nbsp; from&nbsp; the&nbsp; UKM&nbsp; Re- search and Ethics Committee.</p>\r\n<p>\r\n	The&nbsp; subjects were&nbsp; recruited&nbsp; from&nbsp; the Orthodontic&nbsp; Screening Clinic&nbsp; at UKM Dental&nbsp; Polyclinic,&nbsp; over a period of&nbsp; 13 weeks.&nbsp; All patients&nbsp; aged 12 years and above&nbsp; were&nbsp; invited to participate&nbsp; in the study, regardless of gender or ethnicity.&nbsp;</p>\r\n<p>\r\n	Patients younger than 12 years old were not&nbsp; included due to their limited capability to&nbsp; comprehend&nbsp; the information.&nbsp; Instead, one&nbsp; parent&nbsp; for&nbsp; each young patient would take part in the research.&nbsp;&nbsp;</p>\r\n<p>\r\n	When&nbsp; the&nbsp; patients&nbsp; (or&nbsp; parents&nbsp; of&nbsp; young patients)&nbsp; arrived,&nbsp; they&nbsp; were&nbsp; given&nbsp; infor- mation&nbsp; sheets&nbsp; about&nbsp; this&nbsp; research.&nbsp; The patients&nbsp; (or&nbsp; parents&nbsp; of&nbsp; young&nbsp; patients) were informed on the benefits, risks, con- fidentiality&nbsp; involved&nbsp; and&nbsp; participation&nbsp; re- quired.&nbsp; After&nbsp; that,&nbsp; those&nbsp; who&nbsp; agreed&nbsp; to participate&nbsp; would&nbsp; have&nbsp; to&nbsp; complete&nbsp; and return&nbsp; the&nbsp; consent&nbsp; form.&nbsp; Those&nbsp; who&nbsp; de- clined would be interviewed briefly to find out the reason for not doing so.&nbsp;</p>\r\n<p>\r\n	Those&nbsp; who had worn braces&nbsp; before would be excluded because their baseline knowledge&nbsp; was&nbsp; more&nbsp; than the&nbsp; average population.&nbsp; Hence, this&nbsp; group&nbsp; of&nbsp; subjects tended to recall not only information given during&nbsp; the&nbsp; study,&nbsp; but also based on their experience and prior knowledge.</p>\r\n<p>\r\n	Initially,&nbsp; the&nbsp; study&nbsp; was&nbsp; intended&nbsp; to be a randomised&nbsp; control&nbsp; trial.&nbsp; However,&nbsp; during the&nbsp; pre-test,&nbsp; difficulty&nbsp; of&nbsp; randomisation was&nbsp; encountered.&nbsp; Even though patients could be selected randomly using a series of&nbsp; random&nbsp; numbers,&nbsp; overspill&nbsp; of&nbsp; informa- tion&nbsp; occurred due to&nbsp; close&nbsp; proximity&nbsp; of dental chairs. Therefore, convenient sam- pling&nbsp; was chosen&nbsp; in the&nbsp; end, in order to prevent&nbsp; overspill of&nbsp; information&nbsp; which would&nbsp; affect&nbsp; the final&nbsp; result.&nbsp; Patients would be called into one of the two rooms by&nbsp; the&nbsp; Dental&nbsp; Surgical Assistant,&nbsp; without any knowledge of this study. Hence, con- venient sampling was done accordingly.&nbsp; The&nbsp; orthodontic&nbsp; information&nbsp; leaflet&nbsp; was originally&nbsp; produced&nbsp; by&nbsp; the&nbsp; British&nbsp; Ortho- dontic&nbsp; Society.&nbsp; It&nbsp; contained&nbsp; pictorial&nbsp; and textual&nbsp; information,&nbsp; presented&nbsp; under headings designed in a &lsquo;Question and Answer&rsquo;&nbsp; format&nbsp; on a multicoloured,&nbsp; six sec- tion,&nbsp; double-sided&nbsp; A4 sheet. Information included introduction, type of braces, pro- cedure&nbsp; involved,&nbsp; effects,&nbsp; risks&nbsp; and precautions,&nbsp; emergency&nbsp; appointments,&nbsp; retainers,&nbsp; and success rates.&nbsp; In the&nbsp; Malay- sian&nbsp; context,&nbsp; changes were&nbsp; done to the pictures, to portray Asian faces in order to maintain&nbsp; patients&rsquo;&nbsp; acceptability.&nbsp; A questionnaire&nbsp; was developed according&nbsp; to&nbsp; the &lsquo;Question and Answer&rsquo; in the leaflet.</p>\r\n<p>\r\n	The&nbsp; leaflet&nbsp; and the&nbsp; questionnaire were translated&nbsp; into&nbsp; Malay and Chinese&nbsp; lan- guages.&nbsp; The&nbsp; translations&nbsp; were done by the&nbsp; researchers&nbsp; and validated&nbsp; by&nbsp; two qualified&nbsp; translators&nbsp; who had experience translating&nbsp; medical&nbsp; or&nbsp; dental publication materials.&nbsp; Then,&nbsp; validation&nbsp; was repeated by&nbsp; two&nbsp; Orthodontists&nbsp; who were native speakers&nbsp; of&nbsp; Malay and Chinese,&nbsp; respec- tively.&nbsp;</p>\r\n<p>\r\n	The&nbsp; questionnaires&nbsp; were pre-tested&nbsp; on 20&nbsp; screening patients&nbsp; in the&nbsp; beginning of this&nbsp; study.&nbsp; There&nbsp; were no major&nbsp; adjustments apart from the layout and omission of&nbsp; redundant&nbsp; words.&nbsp; Both&nbsp; researchers were&nbsp; able to converse fluently&nbsp; in English, Malay and Chinese.</p>\r\n<p>\r\n	The&nbsp; content&nbsp; of&nbsp; both&nbsp; verbal and written information&nbsp; was exactly&nbsp; the same.&nbsp; The difference&nbsp; was solely&nbsp; the&nbsp; mode&nbsp; of&nbsp; communication.&nbsp; Each participant&nbsp; would be exposed to only ONE type of information, either verbally or in written form, according to&nbsp; their language preference.&nbsp; There&nbsp; were 39 participants in the Verbal group and 40 participants in the Written group.</p>\r\n<p>\r\n	Participants&nbsp; were exposed to the infor- mation;&nbsp; either verbally or in writing for 10 minutes.&nbsp; Participants were not allowed to ask&nbsp; additional questions. Clinicians and accompanying family or friends of the subjects&nbsp; were instructed not to provide help. Those&nbsp; who were given written information were&nbsp; prohibited from&nbsp; keeping the informa- tion&nbsp; leaflet&nbsp; after the exposure time.&nbsp; This was followed by a 15 minutes interval.</p>\r\n<p>\r\n	Self-administered&nbsp; questionnaires&nbsp; were distributed after the 15 minutes interval to assess&nbsp; subject&rsquo;s&nbsp; short-term&nbsp; recall of&nbsp; the information&nbsp; presented.&nbsp; The&nbsp; questionnaire (Appendix 1) was divided into three parts: Part&nbsp; A was on sociodemographic&nbsp; data&nbsp; of the&nbsp; subject. Part B and C were closed questions&nbsp; to&nbsp; assess the&nbsp; subject&rsquo;s&nbsp; level of recall.&nbsp;</p>\r\n<p>\r\n	Part B (Question 1 to 12) covered topics such&nbsp; as indications of&nbsp; orthodontic&nbsp; treat- ment,&nbsp; types&nbsp; of&nbsp; braces,&nbsp; duration&nbsp; of&nbsp; treat- ment,&nbsp; pain experience,&nbsp; frequency&nbsp; of&nbsp; ap- pointments,&nbsp; risks&nbsp; and precautions,&nbsp; emer- gency appointment, retainer, and success rate. Each question was followed by three choices,&nbsp; with&nbsp; only&nbsp; one correct answer. The&nbsp; subject was instructed&nbsp; to circle&nbsp; only one answer.&nbsp;&nbsp;</p>\r\n<p>\r\n	For&nbsp; Part&nbsp; C&nbsp; (Question&nbsp; 13),&nbsp; the&nbsp; subject could&nbsp; choose&nbsp; more&nbsp; than&nbsp; one&nbsp; answer. Such an arrangement was done because it&nbsp; was&nbsp; about&nbsp; the&nbsp; consequences&nbsp; of&nbsp; not taking&nbsp; care&nbsp; of&nbsp; the&nbsp; braces.&nbsp;&nbsp;&nbsp; This&nbsp; informa- tion&nbsp; was&nbsp; of&nbsp; great&nbsp; importance,&nbsp; yet&nbsp; fre- quently&nbsp; ignored&nbsp; by&nbsp; the&nbsp; patients&nbsp; under- going orthodontic treatment.</p>\r\n<p>\r\n	Statistical analysis was conducted using Statistical&nbsp; Package&nbsp; for&nbsp; the&nbsp; Social Sciences&nbsp; (SPSS)&nbsp; version&nbsp; 15.0.&nbsp; Chi- square test was used to compare the pro- portion&nbsp; of&nbsp; correct&nbsp; responses&nbsp; for&nbsp; both&nbsp; ver-bal and written for each question.</p>\r\n<p>\r\n	<strong>RESULTS </strong></p>\r\n<p>\r\n	&nbsp;A&nbsp; total&nbsp; of&nbsp; 89&nbsp; subjects&nbsp; were&nbsp; approached for&nbsp; this&nbsp; study.&nbsp; However,&nbsp; eight&nbsp; of&nbsp; them refused&nbsp; to&nbsp; participate&nbsp; in&nbsp; this&nbsp; study.&nbsp; The commonest&nbsp; reason&nbsp; given&nbsp; was&nbsp; they&nbsp; did not&nbsp; have&nbsp; the&nbsp; time.&nbsp; Two&nbsp; subjects&nbsp; were excluded&nbsp; because&nbsp; they&nbsp; had&nbsp; orthodontic treatment&nbsp; before.&nbsp; The&nbsp; final&nbsp; numbers&nbsp; of subject were 79 in total.&nbsp;</p>\r\n<p>\r\n	The&nbsp; sociodemographic&nbsp; characteristics of&nbsp; the study&nbsp; sample&nbsp; were&nbsp; analysed&nbsp; (Ta- ble&nbsp; 1).&nbsp; The&nbsp; mean&nbsp; age&nbsp; for&nbsp; the verbal group&nbsp; was 20.67,&nbsp; and 21.60 for the written group.&nbsp;</p>\r\n<p>\r\n	A&nbsp; high&nbsp; level&nbsp; of&nbsp; correct&nbsp; responses&nbsp; was received.&nbsp; There&nbsp; were&nbsp; no&nbsp; significant&nbsp; dif- ferences&nbsp; among&nbsp; both groups&nbsp; for&nbsp; all the questions (Table 2 &amp; Table 3).&nbsp;&nbsp;</p>\r\n<p>\r\n	Table 4 showed the information given in Malay,&nbsp; English&nbsp; and&nbsp; Chinese&nbsp; in both&nbsp; ver- bal and written forms.&nbsp;&nbsp;</p>\r\n<p>\r\n	In order to ascertain there was no influ- ence&nbsp; of&nbsp; the&nbsp; language&nbsp; on&nbsp; the&nbsp; results, analysis&nbsp; was&nbsp; done&nbsp; and&nbsp; results&nbsp; showed no&nbsp; significant&nbsp; differences.&nbsp; Therefore,&nbsp; the confounding factor of language used was insignificant (Table 5 &amp; Table 6).&nbsp; DISCUSSION&nbsp; There&nbsp; were&nbsp; more&nbsp; females&nbsp; recruited&nbsp; in this&nbsp; study,&nbsp; as&nbsp; there&nbsp; were&nbsp; more&nbsp; females seeking&nbsp; orthodontic&nbsp; treatment.&nbsp; This&nbsp; is&nbsp; in accordance&nbsp; with&nbsp; a&nbsp; study&nbsp; which&nbsp; stated that&nbsp; girls&nbsp; were&nbsp; more&nbsp; concerned&nbsp; about their&nbsp; smiles&nbsp; and&nbsp; requested&nbsp; orthodontic treatment&nbsp; more&nbsp; often&nbsp; than&nbsp; boys&nbsp; (Chris- topherson&nbsp; et&nbsp; al.&nbsp; 2009).&nbsp; The&nbsp; samples comprised mostly of Malays, followed by Chinese and Indians.&nbsp;</p>\r\n<p>\r\n	Most of the subjects had Sijil Pelajaran Malaysia (SPM) qualification, followed by university&nbsp; graduates.&nbsp; The&nbsp; subjects&nbsp; with Ujian Penilaian Sekolah Rendah (UPSR) qualification came in third, and finally those with Penilaian Menengah Rendah (PMR) qualification.</p>\r\n<p>\r\n	All questions received a high proportion of&nbsp; correct&nbsp; answers.&nbsp; Thomson&nbsp; et al. (2001) suggested such findings could be due&nbsp; to the&nbsp; extra&nbsp; attentiveness&nbsp; of&nbsp; the subjects who were aware that they&nbsp; were taking part in a study.</p>\r\n<p>\r\n	Exception was observed for question 4, which&nbsp; asked&nbsp; whether&nbsp; orthodontic&nbsp; treat- ment is painful. Four subjects of the Ver- bal group and one subject of the Written group&nbsp; gave&nbsp; wrong&nbsp; answers,&nbsp; while&nbsp; two subjects&nbsp; of&nbsp; Written&nbsp; group&nbsp; failed&nbsp; to give any&nbsp; answer.&nbsp; In other&nbsp; words,&nbsp; 7 subjects (8.86%) failed to give the correct answer. This&nbsp; was&nbsp; due&nbsp; to the&nbsp; complexity&nbsp; of&nbsp; the question, which required subjects to think critically.</p>\r\n<p>\r\n	Question&nbsp; 10 received&nbsp; all&nbsp; correct&nbsp; an- swers&nbsp; from&nbsp; both groups.&nbsp; It&nbsp; was&nbsp; a ques- tion&nbsp; about&nbsp; what&nbsp; action&nbsp; to be taken&nbsp; if&nbsp; the orthodontic brace broke during treatment. The&nbsp; question&nbsp; and&nbsp; the correct&nbsp; answer were&nbsp; almost&nbsp; similar&nbsp; to the&nbsp; information given&nbsp; to the&nbsp; subjects.&nbsp; Less&nbsp; thinking&nbsp; was required&nbsp; and&nbsp; all&nbsp; subjects&nbsp; performed&nbsp; well in this question.</p>\r\n<p>\r\n	There&nbsp; were&nbsp; no&nbsp; significant&nbsp; differences between&nbsp; the&nbsp; Verbal&nbsp; and&nbsp; Written&nbsp; groups in&nbsp; all&nbsp; the&nbsp; questions.&nbsp; This&nbsp; finding&nbsp; con- firmed&nbsp; the&nbsp; results&nbsp; of&nbsp; previous&nbsp; study (Thomson et al. 2001).</p>\r\n<p>\r\n	The&nbsp; subject&nbsp; could&nbsp; choose&nbsp; more&nbsp; than one answer in Question 13. The question was&nbsp; about&nbsp; the&nbsp; risks&nbsp; of&nbsp; orthodontic&nbsp; treat- ment. There were a total of three correct answers.&nbsp; Most&nbsp; of&nbsp; the&nbsp; Verbal&nbsp; group (48.7%)&nbsp; gave&nbsp; three&nbsp; correct&nbsp; answers,&nbsp; but most&nbsp; of&nbsp; the&nbsp; Written&nbsp; group&nbsp; (52.5%)&nbsp; only managed&nbsp; to&nbsp; get&nbsp; two&nbsp; correct&nbsp; answers. This&nbsp; could&nbsp; probably&nbsp; be&nbsp; explained&nbsp; by&nbsp; the arrangement of information. Although the sequence&nbsp; of&nbsp; information&nbsp; given&nbsp; was&nbsp; similar&nbsp; for&nbsp; both&nbsp; groups,&nbsp; the&nbsp; written&nbsp; informa- tion had shown more clear-cut partition of information&nbsp; in&nbsp; different&nbsp; paragraphs.&nbsp; The subjects&nbsp; could&nbsp; recall&nbsp; answers&nbsp; from&nbsp; the last&nbsp; paragraph&nbsp; in&nbsp; order&nbsp; to&nbsp; answer&nbsp; this question. However, the risk of &lsquo;permanent scarring&rsquo;&nbsp; could&nbsp; only&nbsp; be&nbsp; found&nbsp; in&nbsp; the&nbsp; ear- lier paragraph. Such arrangement could&nbsp; confuse&nbsp; the subjects&nbsp; with&nbsp; Written&nbsp; infor- mation, but not for the subjects with Ver- bal&nbsp; information.&nbsp; The&nbsp; continuity&nbsp; of&nbsp; the&nbsp; in- formation,&nbsp; and therefore&nbsp; subsequent&nbsp; un- derstanding and recall were in one-piece rather&nbsp; than&nbsp; in&nbsp; partition&nbsp; form.&nbsp; Hence,&nbsp; the succession&nbsp; of&nbsp; one&nbsp; thought&nbsp; to&nbsp; another would not be interrupted.</p>\r\n<p>\r\n	One&nbsp; of&nbsp; the differences&nbsp; between&nbsp; this study&nbsp; and earlier&nbsp; studies&nbsp; was&nbsp; the use&nbsp; of three&nbsp; languages&nbsp; in this&nbsp; study,&nbsp; whereas previous&nbsp; studies&nbsp; only&nbsp; used&nbsp; English&nbsp; (An- derson &amp; Freer 2005; Newton &amp; Thickett 2006; Patel et al. 2008). Therefore, it was mandatory to ascertain that the language used&nbsp; had&nbsp; minimal&nbsp; influence&nbsp; on the&nbsp; re- sults&nbsp; obtained.&nbsp; No&nbsp; significant&nbsp; difference was&nbsp; found&nbsp; between&nbsp; groups&nbsp; given&nbsp; infor- mation in different languages. This, com- pounded&nbsp; with&nbsp; the fact&nbsp; that&nbsp; all translated leaflets&nbsp; were&nbsp; validated&nbsp; by&nbsp; qualified&nbsp; inter- preters&nbsp; and&nbsp; orthodontists,&nbsp; excluded&nbsp; the possibility of the language used resulting in bias in this study.</p>\r\n<p>\r\n	When age, gender, ethnicity, method of information&nbsp; provision&nbsp; and&nbsp; other&nbsp; factors were&nbsp; analysed,&nbsp; Patel&nbsp; et al.&nbsp; (2008)&nbsp; found that only the method of information provision was significant. However, this study showed no significant difference in all the factors&nbsp; listed,&nbsp; even&nbsp; the method&nbsp; of&nbsp; infor- mation&nbsp; provision.&nbsp; Few&nbsp; limitations&nbsp; were recognized&nbsp; during&nbsp; the&nbsp; course&nbsp; of&nbsp; the study,&nbsp; which&nbsp; included&nbsp; sampling&nbsp; method and interval for recall. In order to prevent&nbsp;&nbsp; overspill&nbsp; of&nbsp; information,&nbsp; convenient&nbsp; sampling was selected, but this compromised randomisation&nbsp; of&nbsp; the&nbsp; study.&nbsp; Most&nbsp; of&nbsp; the previous&nbsp; studies&nbsp; compare&nbsp; both&nbsp; short- and long-term recall in order to reflect the memory of the subjects as time went by. However,&nbsp; the&nbsp; time&nbsp; limit&nbsp; of&nbsp; this&nbsp; study&nbsp; did not&nbsp; allow&nbsp; the&nbsp; long-term&nbsp; recall&nbsp; rate&nbsp; to&nbsp; be studied.&nbsp;</p>\r\n<p>\r\n	<strong>CONCLUSION </strong></p>\r\n<p>\r\n	Most&nbsp; of&nbsp; the&nbsp; subjects&nbsp; were&nbsp; able&nbsp; to&nbsp; recall correctly&nbsp; the&nbsp; information&nbsp; that&nbsp; was&nbsp; given. However,&nbsp; no&nbsp; significant&nbsp; difference&nbsp; was found when comparing the level of recall between&nbsp; verbal&nbsp; and&nbsp; written&nbsp; information about orthodontic treatment.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(462, 459, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Cigarette smoking is a major contributing factor&nbsp; of&nbsp; lung&nbsp; cancer,&nbsp; cancers&nbsp; of&nbsp; other sites,&nbsp; chronic&nbsp; obstructive&nbsp; pulmonary&nbsp; dis- ease (COPD) and heart diseases (World Health&nbsp; Organization&nbsp; 2005).&nbsp; In&nbsp; spite&nbsp; of this,&nbsp; smoking&nbsp; prevalence&nbsp; among&nbsp; Malay- sian&nbsp; adults&nbsp; remains&nbsp; high.&nbsp; In&nbsp; 2006,&nbsp; ap- proximately&nbsp; half&nbsp; of&nbsp; male&nbsp; adults&nbsp; aged&nbsp; 18 years and above were smokers (Institute for&nbsp; Public&nbsp; Health&nbsp; 2008).&nbsp; Ten&nbsp; thousand deaths from diseases related to smoking have been reported annually in Malaysia since&nbsp; 1980&nbsp; (Chua&nbsp; 2003).&nbsp; It&nbsp; is&nbsp; estimated that the mortality rate may rise to 30 000 annually&nbsp; by&nbsp; 2020&nbsp; if&nbsp; there&nbsp; is&nbsp; no&nbsp; drastic change&nbsp; in&nbsp; the&nbsp; current&nbsp; trend&nbsp; of&nbsp; smoking among&nbsp; Malaysians&nbsp; (Disease&nbsp; Control&nbsp; Division&nbsp; 2003).&nbsp; The&nbsp; Gateway&nbsp; Drug&nbsp; theory suggests that the use of &lsquo;soft&rsquo; drugs such as cigarettes can lead to the use of other &lsquo;harder&rsquo; drugs (Fleming et al. 1989). Pre- vious&nbsp; studies&nbsp; have&nbsp; also&nbsp; shown&nbsp; that smokers&nbsp; are&nbsp; more&nbsp; likely&nbsp; to&nbsp; risk&nbsp; harming themselves&nbsp; further&nbsp; by&nbsp; resorting&nbsp; to&nbsp; other abusive&nbsp; substances&nbsp; compared&nbsp; to&nbsp; non- smokers&nbsp; (Fleming&nbsp; et&nbsp; al.&nbsp; 1989;&nbsp; Kandel&nbsp; et al.&nbsp; 1992).&nbsp; Therefore,&nbsp; at&nbsp; this&nbsp; critical&nbsp; mo- ment,&nbsp; it&nbsp; comes&nbsp; as&nbsp; no&nbsp; surprise&nbsp; that&nbsp; re- ducing&nbsp; the&nbsp; prevalence&nbsp; and&nbsp; incidence&nbsp; of smoking&nbsp; is&nbsp; a priority&nbsp; of&nbsp; the&nbsp; Ministry&nbsp; of Health. Studies have shown that smoking is&nbsp; a learned&nbsp; behavior&nbsp; which&nbsp; generally starts&nbsp; during&nbsp; adolescence.&nbsp; It has&nbsp; been reported that 80-90% of current smokers start&nbsp; smoking&nbsp; as&nbsp; adolescents,&nbsp; and&nbsp; be- come&nbsp; regular&nbsp; smokers&nbsp; before&nbsp; the age of 18&nbsp; (USDHHS&nbsp; 1994;&nbsp; Health&nbsp; Canada 2005). Adolescents who smoke are either unwilling to quit smoking, have low moti- vation&nbsp; to&nbsp; quit&nbsp; or&nbsp; are&nbsp; uninterested&nbsp; to quit because&nbsp; they&nbsp; do&nbsp; not&nbsp; think&nbsp; quitting&nbsp; is&nbsp; im- portant&nbsp; (Balch&nbsp; 1998;&nbsp; Pallonen&nbsp; 1998; Mermelstein&nbsp; 2003).&nbsp; These&nbsp; factors&nbsp; have driven researchers to focus on strategies, at&nbsp; the earliest&nbsp; possible&nbsp; opportunity,&nbsp; at identifying&nbsp; those&nbsp; adolescents&nbsp; who&nbsp; face the&nbsp; greatest&nbsp; risk&nbsp; of&nbsp; becoming&nbsp; habitual smokers&nbsp; in the future.&nbsp; Such&nbsp; a move&nbsp; will certainly&nbsp; help&nbsp; smoking-prevention&nbsp; pro- grammes to be targeted to this group and hopefully,&nbsp; reduce&nbsp; smoking&nbsp; initiation among them.</p>\r\n<p>\r\n	A&nbsp; &lsquo;susceptibility-to-smoking&rsquo;&nbsp; measure has&nbsp; been developed&nbsp; to identify&nbsp; adoles- cents who are at risk of initiating smoking (Pierce et al. 1996). It measures the cog- nitive&nbsp; commitment&nbsp; of&nbsp; adolescents&nbsp; not to smoke&nbsp; and adolescents&rsquo;&nbsp; capability&nbsp; to resist offers from friends to smoke. Unger et&nbsp; al.&nbsp; (1997)&nbsp; and&nbsp; Difranza&nbsp; et al.&nbsp; (2006)</p>\r\n<p>\r\n	reported that respondents who were sus- ceptible&nbsp; to&nbsp; smoking&nbsp; were&nbsp; two&nbsp; to&nbsp; three times&nbsp; at&nbsp; higher&nbsp; risk&nbsp; of&nbsp; becoming&nbsp; experi- mental&nbsp; smokers&nbsp; after&nbsp; a&nbsp; period&nbsp; of&nbsp; two years. Jackson (1998) revealed that cog- nitive&nbsp; susceptibility&nbsp; was&nbsp; the&nbsp; most&nbsp; signifi- cant&nbsp; predictor&nbsp; of&nbsp; smoking&nbsp; initiation among&nbsp; elementary&nbsp; school&nbsp; students&nbsp; after one year. Those who were susceptible to smoking were 80% more likely to&nbsp; initiate smoking&nbsp; compared&nbsp; to&nbsp; non-susceptible respondents.&nbsp; Cognitive&nbsp; susceptibility&nbsp; has been&nbsp; reported&nbsp; to&nbsp; be&nbsp; a&nbsp; more&nbsp; significant independent&nbsp; predictor&nbsp; of&nbsp; experimental smoking&nbsp; compared&nbsp; to&nbsp; parental&nbsp; smoking and&nbsp; peer&nbsp; smoking&nbsp; (Pierce&nbsp; et&nbsp; al.&nbsp; 1996). Huang&nbsp; et&nbsp; al&nbsp; (2005)&nbsp; in&nbsp; their&nbsp; longitudinal study&nbsp; among&nbsp; adolescents&nbsp; aged&nbsp; from&nbsp; 14 to&nbsp; 17&nbsp; years&nbsp; old&nbsp; found&nbsp; that&nbsp; susceptible adolescents were also two to three times more&nbsp; likely&nbsp; to&nbsp; initiate&nbsp; smoking&nbsp; after&nbsp; two years.&nbsp; The&nbsp; simplicity&nbsp; of&nbsp; the&nbsp; measure lends&nbsp; itself&nbsp; as&nbsp; a&nbsp; useful&nbsp; and&nbsp; efficient&nbsp; pre- dictor&nbsp; of&nbsp; smoking&nbsp; initiation.&nbsp; Although&nbsp; the &ldquo;susceptibility&rdquo; measure has been shown to be an effective tool for identifying and predicting&nbsp; adolescent&nbsp; smoking&nbsp; initiation, it&nbsp; has&nbsp; not,&nbsp; hitherto,&nbsp; been&nbsp; applied&nbsp; in&nbsp; Ma- laysia.&nbsp; The&nbsp; aims&nbsp; of&nbsp; this&nbsp; study&nbsp; were&nbsp; to determine the validity of the measure and to&nbsp; identify&nbsp; the&nbsp; independent&nbsp; factors&nbsp; for smoking&nbsp; initiation&nbsp; among&nbsp; school&nbsp; children in Kota Tinggi district, Johor, Malaysia.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS </strong></p>\r\n<p>\r\n	Study Design&nbsp;</p>\r\n<p>\r\n	This&nbsp; was&nbsp; a&nbsp; longitudinal&nbsp; study&nbsp; conducted from&nbsp; 2008&nbsp; to&nbsp; 2009.&nbsp; Baseline&nbsp; data&nbsp; was collected&nbsp; in&nbsp; May&nbsp; 2008&nbsp; and&nbsp; follow&nbsp; up&nbsp; in June&nbsp; 2009.&nbsp; Respondents&nbsp; were&nbsp; second- ary school students&nbsp; in Forms 1, 2&nbsp; and 4 in the district of Kota Tinggi in Johor. The study&nbsp; was&nbsp; approved&nbsp; by&nbsp; the&nbsp; Ministry&nbsp; of Education&nbsp; Malaysia&nbsp; and&nbsp; the&nbsp; Johor&nbsp; State Education&nbsp; Department&nbsp; while&nbsp; ethics&nbsp; ap- proval&nbsp; was&nbsp; given&nbsp; by&nbsp; the&nbsp; Ethics&nbsp; commit- tee,&nbsp; Ministry&nbsp; of&nbsp; Health,&nbsp; Malaysia.&nbsp; This project was a collaborative effort between the&nbsp; Institute&nbsp; for&nbsp; Medical&nbsp; Research&nbsp; (IMR) and the Kota Tinggi District Health Office.&nbsp;</p>\r\n<p>\r\n	Study protocol&nbsp;</p>\r\n<p>\r\n	Passive&nbsp; consent&nbsp; was&nbsp; obtained&nbsp; from&nbsp; stu- dents&rsquo;&nbsp; parents&nbsp; prior&nbsp; to their&nbsp; participation in the study. Respondents were informed that participation was voluntary. Students who&nbsp; agreed&nbsp; to participate&nbsp; were asked to put&nbsp; down&nbsp; their&nbsp; signatures&nbsp; on the ques- tionnaire&nbsp; and&nbsp; were&nbsp; given&nbsp; assurance&nbsp; of anonymity. In addition, teachers were not allowed&nbsp; to observe&nbsp; the&nbsp; students&nbsp; while they were completing the questionnaires. The&nbsp; questionnaires&nbsp; were&nbsp; self-adminis- tered&nbsp; by&nbsp; the&nbsp; students.&nbsp; A detailed&nbsp; expla- nation on the questionnaire was given to the&nbsp; respondents.&nbsp; Further&nbsp; clarification&nbsp; on any of the items was given when needed. At&nbsp; the&nbsp; end&nbsp; of&nbsp; the&nbsp; session,&nbsp; completed questionnaires&nbsp; were&nbsp; packed&nbsp; into&nbsp; enve- lopes,&nbsp; and the&nbsp; envelopes&nbsp; sealed&nbsp; in the presence of the respondents.</p>\r\n<p>\r\n	Sampling&nbsp;</p>\r\n<p>\r\n	Multi-stage&nbsp; sampling&nbsp; was&nbsp; used&nbsp; in&nbsp; this study.&nbsp; Schools&nbsp; were&nbsp; stratified&nbsp; by&nbsp; urban, rural&nbsp; and&nbsp; FELDA&nbsp; settlement&nbsp; areas.&nbsp; Six schools&nbsp; were&nbsp; randomly&nbsp; selected&nbsp; from FELDA settlement areas, three from town areas&nbsp; and&nbsp; one&nbsp; from&nbsp; a&nbsp; rural&nbsp; area.&nbsp; Stu- dents&nbsp; in&nbsp; the&nbsp; selected&nbsp; schools&nbsp; were&nbsp; then stratified&nbsp; by&nbsp; Forms:&nbsp; Form 1,&nbsp; Form&nbsp; 2&nbsp; and Form&nbsp; 4.&nbsp; The&nbsp; sampling&nbsp; frame&nbsp; was&nbsp; then obtained&nbsp; from&nbsp; the&nbsp; schools&rsquo;&nbsp; administra- tors,&nbsp; and&nbsp; simple&nbsp; random&nbsp; sampling&nbsp; was used to select respondents using random numbers&nbsp; generated&nbsp; by&nbsp; Epi&nbsp; Info&nbsp; version 6.04d.</p>\r\n<p>\r\n	A&nbsp; sample&nbsp; size&nbsp; of&nbsp; 2700&nbsp; was&nbsp; calculated based on smoking incidence of 3.5% for Forms&nbsp; 1 and 2, and 6%&nbsp; for&nbsp; Form&nbsp; 4 stu- dents,&nbsp; maximum&nbsp; error&nbsp; of 3%,&nbsp; design&nbsp; ef- fect of 0.67, intra-class correlation coeffi- cient&nbsp; of&nbsp; 0.5,&nbsp; average&nbsp; proportion&nbsp; of&nbsp; stu- dents&nbsp; per&nbsp; strata&nbsp; of&nbsp; 0.33&nbsp; (prevalence&nbsp; of smoking&nbsp; was&nbsp; assumed&nbsp; to&nbsp; be&nbsp; equal among&nbsp; all&nbsp; three&nbsp; strata),&nbsp; an&nbsp; additional 30% extra to cater for non response and another&nbsp; 30%&nbsp; for&nbsp; exclusion&nbsp; of&nbsp; smokers. (Kalton&nbsp; 1983;&nbsp; Scheaffer&nbsp; et&nbsp; al.&nbsp; 1990; United Nation 2005). The number of stu- dents selected from each school was de- rived as a&nbsp; proportion to the total student population&nbsp; in&nbsp; the&nbsp; respective&nbsp; schools. Students&nbsp; who&nbsp; were&nbsp; non-smokers&nbsp; at baseline were followed up after one year. Each student was given a unique identifi- cation&nbsp; (ID)&nbsp; number&nbsp; at&nbsp; the&nbsp; beginning&nbsp; of the study, and the same ID was allocated to the student during the follow-up study.</p>\r\n<p>\r\n	Study instrument</p>\r\n<p>\r\n	&nbsp;The&nbsp; same&nbsp; self-administered&nbsp; question- naire was used at baseline and at follow- up. The instrument used in this study was adapted&nbsp; from&nbsp; previous&nbsp; studies&nbsp; (Hanjeet et&nbsp; al.&nbsp; 2001;&nbsp; Lim&nbsp; et&nbsp; al.&nbsp; 2006)&nbsp; and&nbsp; the questionnaire&nbsp; included&nbsp; demographic&nbsp; and psychosocial&nbsp; factors.&nbsp; These&nbsp; were:&nbsp; per- centage&nbsp; of&nbsp; peers&nbsp; who&nbsp; smoke,&nbsp; family members&nbsp; who&nbsp; smoke,&nbsp; percentage&nbsp; of friends&nbsp; who&nbsp; smoke,&nbsp; and&nbsp; perceptions&nbsp; of both&nbsp; parental&nbsp; and&nbsp; social&nbsp; acceptance&nbsp; of smoking.&nbsp; Perceptions&nbsp; of&nbsp; parental&nbsp; and social&nbsp; acceptance&nbsp; of&nbsp; smoking&nbsp; were&nbsp; as- sessed&nbsp; using&nbsp; a&nbsp; 7-point&nbsp; Likert&nbsp; type&nbsp; scale in&nbsp; which&nbsp; lower&nbsp; scores&nbsp; indicated&nbsp; lower acceptance&nbsp; by&nbsp; parents&nbsp; and&nbsp; society&nbsp; to- wards&nbsp; smoking&nbsp; behavior&nbsp; among&nbsp; adoles- cents. Perceived benefits of smoking and perceived&nbsp; negative&nbsp; effects&nbsp; of&nbsp; smoking were&nbsp; assessed&nbsp; using&nbsp; 12&nbsp; questions&nbsp; com- prising&nbsp; six&nbsp; questions&nbsp; for&nbsp; each.&nbsp; Low scores&nbsp; in&nbsp; the&nbsp; &ldquo;Perceived&nbsp; benefits&nbsp; of smoking&rdquo;&nbsp; domain&nbsp; indicate&nbsp; positive&nbsp; per- ception toward smoking, while low scores in&nbsp; the&nbsp; &ldquo;Perceived&nbsp; negative&nbsp; effects&nbsp; of smoking&rdquo; domain indicate highly negative perception&nbsp; of&nbsp; respondents&nbsp; towards smoking.&nbsp; The&nbsp; &lsquo;Susceptibility-to-Smoking&rsquo; measure&nbsp; was&nbsp; adapted&nbsp; from&nbsp; Pierce&nbsp; et&nbsp; al. (1996). It consisted of two questions:</p>\r\n<p>\r\n	a)&nbsp;&nbsp; Do you think you will smoke a ciga- rette in the next year?<br />\r\n	b)&nbsp;&nbsp; If&nbsp; one of&nbsp; your&nbsp; best&nbsp; friends&nbsp; were&nbsp; to offer&nbsp; you a cigarette,&nbsp; would&nbsp; you smoke?</p>\r\n<p>\r\n	&nbsp;The choice of answers was: (a) Yes, (b) Probably&nbsp; yes,&nbsp; (c)&nbsp; Probably&nbsp; no, and (d) Not&nbsp; at all.&nbsp; If&nbsp; the respondent&nbsp; answered &lsquo;Not&nbsp; at all&rsquo;&nbsp; to both questions,&nbsp; they&nbsp; were categorised as &lsquo;Not susceptible to smok- ing&rsquo; while those who gave other answers to&nbsp; both&nbsp; questions&nbsp; were&nbsp; categorised&nbsp; as &lsquo;Susceptible to smoking&rsquo;.</p>\r\n<p>\r\n	The&nbsp; dependent&nbsp; variable&nbsp; in&nbsp; this&nbsp; study was&nbsp; smoking&nbsp; initiation,&nbsp; with&nbsp; &lsquo;Yes&rsquo;&nbsp; or&nbsp; &lsquo;No&rsquo; responses, which&nbsp; was measured at one- year&nbsp; follow-up.&nbsp; Smoking&nbsp; initiation&nbsp; was defined as &lsquo;Have smoked at least once in the last 30 days&rsquo;.&nbsp;</p>\r\n<p>\r\n	Statistical analysis</p>\r\n<p>\r\n	&nbsp;Data&nbsp; were&nbsp; entered&nbsp; and&nbsp; analysed&nbsp; using SPSS&nbsp; version&nbsp; 16&nbsp; (SPSS,&nbsp; 2007).&nbsp; Chi square&nbsp; and&nbsp; independent&nbsp; t&nbsp; test&nbsp; were&nbsp; em- ployed&nbsp; in&nbsp; attrition&nbsp; analysis.&nbsp; The&nbsp; attrition analysis&nbsp; was&nbsp; done&nbsp; to&nbsp; determine&nbsp; the&nbsp; ex- tent&nbsp; to&nbsp; which&nbsp; respondents&nbsp; who&nbsp; dropped out of the study differed from the respon- dents who continued to participate at fol- low&nbsp; up.&nbsp; These&nbsp; tests&nbsp; were&nbsp; also&nbsp; used&nbsp; for associations&nbsp; between&nbsp; categorical&nbsp; variables&nbsp; and&nbsp; continuous&nbsp; data&nbsp; with&nbsp; the&nbsp; de- pendent&nbsp; variable&nbsp; that&nbsp; is&nbsp; initiation&nbsp; of smoking after one year. Independent va- riables&nbsp; are&nbsp; gender,&nbsp; susceptible&nbsp; to&nbsp; smoking,&nbsp; percentage&nbsp; of&nbsp; friends&nbsp; who&nbsp; smoked, form&nbsp; of&nbsp; study,&nbsp; schooling&nbsp; area,&nbsp; father smoking,&nbsp; parents&rsquo;&nbsp; reaction&nbsp; toward&nbsp; smoking,&nbsp; perceived&nbsp; benefits&nbsp; of&nbsp; smoking&nbsp; and perceived&nbsp; negative&nbsp; effects&nbsp; of&nbsp; smoking. Variables with p value equal or less than 0.25&nbsp; from&nbsp; the&nbsp; univariate&nbsp; analysis&nbsp; were included into binary logistic regression to determine the independence of susceptibility-to-smoking&nbsp; variable&nbsp; from&nbsp; other&nbsp; predictor&nbsp; variables.&nbsp; Analysis&nbsp; for&nbsp; two&nbsp; way&nbsp; interactions&nbsp; reveals&nbsp; a&nbsp; non-significant&nbsp; inte- raction&nbsp; between&nbsp; all&nbsp; the&nbsp; variables&nbsp; in&nbsp; the model.&nbsp; Model&nbsp; fit&nbsp; was&nbsp; checked&nbsp; using Hosmer-Lemeshow&nbsp; goodness&nbsp; of&nbsp; fit&nbsp; test. The p value was not significant indicating the&nbsp; model&nbsp; fitted&nbsp; the&nbsp; data&nbsp; (p&nbsp; =&nbsp; 0.640).&nbsp; The&nbsp; level&nbsp; of&nbsp; significance&nbsp; was&nbsp; set&nbsp; at&nbsp; p- value of less than 0.05 (2-sided).&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS&nbsp; </strong></p>\r\n<p>\r\n	Table 1 shows&nbsp; that, out of the 1763 non smokers&nbsp; at&nbsp; baseline&nbsp; in&nbsp; 2008,&nbsp; 1288 (73.1%) responded at follow -up in 2009. Attrition&nbsp; analysis&nbsp; revealed&nbsp; that&nbsp; three&nbsp; in- dependent&nbsp; variables,&nbsp; i.e.&nbsp; form,&nbsp; gender and&nbsp; locality,&nbsp; were&nbsp; significantly&nbsp; different between those who responded at follow- up&nbsp; and&nbsp; those&nbsp; who&nbsp; dropped&nbsp; out.&nbsp; Attrition was higher among female (29.1%), Form Four&nbsp; students&nbsp; (30.7%)&nbsp; and&nbsp; FELDA&nbsp; set- tlement areas (29.5%)&nbsp;</p>\r\n<p>\r\n	From the univariate analysis, these&nbsp; va- riables were associated with the initiation of&nbsp; smoking:&nbsp; percentages&nbsp; of&nbsp; friends&nbsp; who smoke,&nbsp; father&rsquo;s&nbsp; smoking&nbsp; status,&nbsp; suscep- tibility-to&nbsp; smoking,&nbsp; and&nbsp; gender.&nbsp; Gender and&nbsp; susceptibility-to-smoking&nbsp; were&nbsp; the most&nbsp; significant&nbsp; independent&nbsp; variables associated with smoking initiation, that is, at&nbsp; 21.7%&nbsp; for&nbsp; male&nbsp; respondents,&nbsp; com- pared&nbsp; to&nbsp; females&nbsp; at&nbsp; 2.4%.&nbsp; Respondents who&nbsp; were&nbsp; susceptible&nbsp; were&nbsp; approx- imately&nbsp; four&nbsp; times&nbsp; more&nbsp; likely&nbsp; to&nbsp; initiate smoking&nbsp; compared&nbsp; to&nbsp; their&nbsp; counterparts who were not susceptible, that is, 31.9% as compared to 8.2 %. It also shows that initiators, firstly, believed more strongly in the&nbsp; perceived&nbsp; positive&nbsp; consequences&nbsp; of smoking,&nbsp; however&nbsp; misguided&nbsp; this&nbsp; may be,&nbsp; secondly,&nbsp; perceived&nbsp; more&nbsp; accep- tance&nbsp; of&nbsp; parents&nbsp; towards&nbsp; their&nbsp; smoking behavior,&nbsp; and&nbsp; thirdly,&nbsp; perceived&nbsp; less negative&nbsp; consequences&nbsp; of&nbsp; smoking.&nbsp; The two groups were not significantly different on&nbsp; opinions&nbsp; of&nbsp; (societal)&nbsp; perceptions&nbsp; to- wards&nbsp; adolescents&nbsp; smoking&nbsp; (Table&nbsp; 2). Table&nbsp; 3&nbsp; shows&nbsp; the&nbsp; results&nbsp; of multiple&nbsp; lo- gistic&nbsp; regression&nbsp; analysis.&nbsp; Susceptibility to&nbsp; smoking&nbsp; remained&nbsp; a&nbsp; significant&nbsp; risk factor for smoking initiation after a period of&nbsp; one&nbsp; year,&nbsp; after&nbsp; controlling&nbsp; for&nbsp; the&nbsp; po- tential&nbsp; confounding&nbsp; effects&nbsp; of&nbsp; gender, school locality, percentage of friends who smoke,&nbsp; father&nbsp; smoking,&nbsp; parental&nbsp; accep- tance&nbsp; of smoking,&nbsp; and belief in the posi- tive&nbsp; and&nbsp; negative&nbsp; consequences&nbsp; of smoking.&nbsp; The&nbsp; odds&nbsp; ratio&nbsp; was,&nbsp; however, attenuated&nbsp; by&nbsp; the&nbsp; inclusion&nbsp; of&nbsp; the&nbsp; other independent variables.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	&nbsp;To&nbsp; our&nbsp; knowledge,&nbsp; this&nbsp; is&nbsp; the&nbsp; first&nbsp; publi- cation&nbsp; towards&nbsp; establishing&nbsp; &lsquo;Susceptibil- ity-to-Smoking&rsquo; as a reliable predictor for identifying&nbsp; adolescents&nbsp; who&nbsp; have&nbsp; the potential&nbsp; to&nbsp; initiate&nbsp; smoking&nbsp; in&nbsp; Malaysia. The&nbsp; response&nbsp; rate&nbsp; was&nbsp; almost&nbsp; similar&nbsp; to previous&nbsp; longitudinal&nbsp; studies&nbsp; (Conrad&nbsp; et al.&nbsp; 1992).&nbsp; The&nbsp; study&nbsp; revealed&nbsp; that&nbsp; this measure is a reliable predictor for identi- fying&nbsp; adolescents&nbsp; who&nbsp; are&nbsp; at&nbsp; risk&nbsp; of smoking initiation, as well as, for predict- ing&nbsp; smoking&nbsp; initiation&nbsp; among&nbsp; non- smokers at baseline. It shows a substan- tial&nbsp; effect&nbsp; size&nbsp; and&nbsp; it&nbsp; predicts&nbsp; smoking initiation&nbsp; independently&nbsp; of&nbsp; other&nbsp; predic- tors&nbsp; such&nbsp; as,&nbsp; peer&nbsp; smoking,&nbsp; perceived benefits and negative effects of smoking, family&nbsp; members&nbsp; (parents&nbsp; and&nbsp; sibling) smoking,&nbsp; social&nbsp; norms&nbsp; of&nbsp; smoking, gender&nbsp; and&nbsp; age,&nbsp; and&nbsp; factors&nbsp; that&nbsp; have been&nbsp; shown&nbsp; to&nbsp; contribute&nbsp; to&nbsp; smoking&nbsp; in previous&nbsp; studies&nbsp; (Kalton&nbsp; 1983;&nbsp; Kremers et&nbsp; al.&nbsp; 2001;&nbsp; Maxwell&nbsp; 2002;&nbsp; Wen&nbsp; et&nbsp; al. 2005;&nbsp; Wilkinson&nbsp; et&nbsp; al.&nbsp; 2007;&nbsp; Otten&nbsp; et&nbsp; al. 2009)&nbsp; Almost&nbsp; a&nbsp; third&nbsp; of&nbsp; the&nbsp; adolescents who&nbsp; did&nbsp; not&nbsp; smoke&nbsp; but&nbsp; were&nbsp; susceptible to&nbsp; smoking&nbsp; at&nbsp; baseline&nbsp; initiated&nbsp; smoking after a period of one year.</p>\r\n<p>\r\n	Susceptible adolescents were 3.7 times more&nbsp; likely&nbsp; to&nbsp; be smokers&nbsp; at follow-up. This finding is similar to that reported by Pierce&nbsp; et&nbsp; al. (1996),&nbsp; Unger&nbsp; et al. (1997) and&nbsp; Huang&nbsp; et&nbsp; al.&nbsp; (2005).&nbsp; The&nbsp; effect&nbsp; size of&nbsp; the &lsquo;Susceptibility-to-Smoking&rsquo;&nbsp; meas- ure&nbsp; was&nbsp; attenuated&nbsp; after&nbsp; other&nbsp; in dependent variables were included in the multivariate&nbsp; analysis&nbsp; model.&nbsp; This&nbsp; sug- gests&nbsp; that&nbsp; a&nbsp; better&nbsp; model&nbsp; may&nbsp; be&nbsp; devel- oped from a combination of this construct with&nbsp; other&nbsp; predictor&nbsp; variables&nbsp; such&nbsp; as gender and smoking status of fathers.</p>\r\n<p>\r\n	These&nbsp; findings&nbsp; show&nbsp; that&nbsp; the&nbsp; measure can&nbsp; be used&nbsp; in adolescent&nbsp; prevention programmes to reduce smoking initiation among&nbsp; adolescents&nbsp; in the&nbsp; long&nbsp; term. Needless&nbsp; to&nbsp; say,&nbsp; co-operation&nbsp; between school and health authorities is essential, if not vital, to the successful implementa- tion&nbsp; of&nbsp; an effective&nbsp; long&nbsp; term&nbsp; strategy&nbsp; to reduce&nbsp; smoking&nbsp; initiation&nbsp; among&nbsp; our youths.&nbsp; School&nbsp; authorities&nbsp; can&nbsp; carry&nbsp; out surveys using the construct to determine the&nbsp; susceptibility&nbsp; level&nbsp; among&nbsp; those&nbsp; who are&nbsp; not smoking&nbsp; and at the&nbsp; same&nbsp; time, health&nbsp; departments&nbsp; can&nbsp; play&nbsp; a role&nbsp; as training&nbsp; providers.&nbsp; The&nbsp; activities&nbsp; that&nbsp; are to be carried out should be based on the susceptibility level of the adolescent. Dis- semination&nbsp; of&nbsp; information&nbsp; on&nbsp; the&nbsp; health hazards of smoking and training to incul- cate&nbsp; the&nbsp; ability&nbsp; to&nbsp; resist&nbsp; smoking&nbsp; offers from&nbsp; peers&nbsp; should&nbsp; be&nbsp; delivered&nbsp; to&nbsp; non- susceptible&nbsp; groups&nbsp; (Johnson&nbsp; 1990).&nbsp; The formation of peer support groups in order to&nbsp; develop&nbsp; the&nbsp; required&nbsp; attitude&nbsp; towards the smoking&nbsp; habit&nbsp; among those&nbsp; who are susceptible&nbsp; is&nbsp; recommended.&nbsp; Care should be taken to prevent stigmatisation of&nbsp; those&nbsp; who&nbsp; have&nbsp; been&nbsp; identified&nbsp; as susceptible&nbsp; to&nbsp; initiate&nbsp; smoking&nbsp; and&nbsp; also to&nbsp; prevent&nbsp; self-fulfilling&nbsp; prophecies&nbsp; that trigger&nbsp; adolescents&nbsp; to&nbsp; initiate&nbsp; smoking (Unger et al. 1997).</p>\r\n<p>\r\n	The&nbsp; measure&nbsp; can&nbsp; also&nbsp; be&nbsp; used&nbsp; by health&nbsp; workers&nbsp; at&nbsp; adolescent&nbsp; health&nbsp; clin-ics&nbsp; to&nbsp; identify&nbsp; susceptible&nbsp; adolescents, where&nbsp; appropriate&nbsp; advice&nbsp; and&nbsp; proactive counseling&nbsp; can&nbsp; be&nbsp; given&nbsp; to&nbsp; reduce&nbsp; their risk&nbsp; of&nbsp; initiating&nbsp; smoking&nbsp; in&nbsp; the&nbsp; future. Adolescents&nbsp; who&nbsp; are&nbsp; not&nbsp; susceptible&nbsp; to smoking&nbsp; should&nbsp; be&nbsp; given&nbsp; advice&nbsp; and support&nbsp; against&nbsp; smoking,&nbsp; including&nbsp; tips and&nbsp; methods&nbsp; on&nbsp; how&nbsp; to&nbsp; improve&nbsp; their refusal skills. A study showed that advice given by health professionals regarding a health&nbsp; subject&nbsp; is&nbsp; more&nbsp; acceptable&nbsp; and effective&nbsp; for&nbsp; attitudinal&nbsp; behavioural changes (Morgan et al. 1996).</p>\r\n<p>\r\n	There was a limitation in this study. The high&nbsp; attrition&nbsp; of&nbsp; respondents&nbsp; after&nbsp; a pe- riod of one year may reduce&nbsp; the internal and&nbsp; external&nbsp; consistency&nbsp; of&nbsp; the findings. Differential&nbsp; attrition&nbsp; (more&nbsp; females,&nbsp; Form Four&nbsp; students&nbsp; and FELDA&nbsp; settlement areas&nbsp; dropped&nbsp; out)&nbsp; indicated&nbsp; that&nbsp; the follow-up&nbsp; sample&nbsp; may&nbsp; not&nbsp; represent&nbsp; the population of interest which is all non-cur- rent&nbsp; smoking&nbsp; secondary&nbsp; school&nbsp; students in the district.&nbsp;</p>\r\n<p>\r\n	The study reveals that the susceptibility measure&nbsp; was&nbsp; a significant&nbsp; independent predictor&nbsp; to smoking&nbsp; initiation&nbsp; among adolescents.&nbsp; It may&nbsp; also&nbsp; be used&nbsp; as&nbsp; a screening&nbsp; instrument&nbsp; to identify&nbsp; those adolescents&nbsp; who do&nbsp; not smoke&nbsp; but who are at increased risk to smoking initiation.</p>\r\n<p>\r\n	School&nbsp; personnel especially&nbsp; those in the secondary&nbsp; sector&nbsp; can&nbsp; use&nbsp; this&nbsp; measure to identify adolescents who are at risk of initiating&nbsp; smoking&nbsp; as&nbsp; the&nbsp; very&nbsp; first&nbsp; pre- ventive step towards battling the smoking scourge.&nbsp; The&nbsp; use&nbsp; of&nbsp; the&nbsp; measure&nbsp; might reduce&nbsp; the&nbsp; incidence&nbsp; of&nbsp; smoking&nbsp; among our&nbsp; adolescents,&nbsp; and&nbsp; ultimately&nbsp; contri- bute&nbsp; to&nbsp; lowering&nbsp; morbidity&nbsp; and&nbsp; mortality resulting from smoking-related diseases.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS&nbsp; </strong></p>\r\n<p>\r\n	We would like to thank the Director-Gen- eral of Health Malaysia for his permission to publish this paper. We would also like to&nbsp; thank&nbsp; the&nbsp; Ministry&nbsp; of&nbsp; Education,&nbsp; the schools&nbsp; involved&nbsp; in&nbsp; the&nbsp; study&nbsp; and&nbsp; all those who assisted in data collection and management&nbsp; for&nbsp; their&nbsp; support&nbsp; and&nbsp; cooperation.</p>\r\n', 1),
(463, 460, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Cancer of the cervix is an important public&nbsp; health&nbsp; problem&nbsp; worldwide&nbsp; and&nbsp; is&nbsp; the second&nbsp; most&nbsp; common&nbsp; cancer&nbsp; among women&nbsp; (Woodman&nbsp; et&nbsp; al.&nbsp; 2007).&nbsp; In&nbsp; Malaysia,&nbsp; it&nbsp; is&nbsp; the&nbsp; second&nbsp; most&nbsp; common cancer among women in all major ethnic groups&nbsp; (Ministry&nbsp; of&nbsp; Health&nbsp; Malaysia 2003). Human papilloma virus (HPV) has been&nbsp; identified&nbsp; as&nbsp; the most&nbsp; important&nbsp; viral group associated with benign and ma- lignant neoplasia in humans; HPV DNA is estimated to be present in over 99.7% of these&nbsp; cancers&nbsp; (Walboomers&nbsp; et&nbsp; al.&nbsp; 1999; Lockwood&nbsp; &amp;&nbsp; Mcintyre&nbsp; 2009).&nbsp; Using&nbsp; real time PCR method, Panu et al. (2002) and Bourgo&nbsp; et&nbsp; al.&nbsp; (2008)&nbsp; showed&nbsp; the&nbsp; presence of integrated HPV type 16&nbsp; in cervical&nbsp; Intraepithelial&nbsp; neoplasia&nbsp; (CIN).&nbsp; Inte- gration&nbsp; of&nbsp; viral&nbsp; genome&nbsp; into&nbsp; the&nbsp; nucleus has been proposed as the mechanism for activation,&nbsp; proliferation&nbsp; and&nbsp; progression from&nbsp; non-invasive&nbsp; lesions&nbsp; to cervical cancers (Mitsuo et al. 1999; Fan &amp; Chen 2004;&nbsp; Lockwood&nbsp; &amp; Mcintyre&nbsp; 2009).&nbsp; The Hybrid&nbsp; Capture&nbsp; (HC)&nbsp; assay,&nbsp; the&nbsp; current diagnostic test for the screening of cervical&nbsp; cancer,&nbsp; characterizes&nbsp; HPV&nbsp; infections as&nbsp; low&nbsp; or&nbsp; medium/high&nbsp; risk&nbsp; but fails&nbsp; to identify&nbsp; HPV&nbsp; infections&nbsp; by&nbsp; type&nbsp; (Sonnex 1998; Monica et al. 2002 &amp; Seaman et al. 2010).&nbsp; Real&nbsp; time&nbsp; PCR&nbsp; is&nbsp; an efficient technique&nbsp; that&nbsp; objectively&nbsp; detects&nbsp; and quantifies DNA target sequence in a period of two or three hours, whereas classical&nbsp; PCR&nbsp; needs&nbsp; at&nbsp; least&nbsp; four&nbsp; hours&nbsp; to detect the same sequence without quantifying it (Bettini et al. 2008).&nbsp;&nbsp;</p>\r\n<p>\r\n	Recently,&nbsp; two&nbsp; vaccines&nbsp; were made available&nbsp; for&nbsp; protection&nbsp; of&nbsp; infection&nbsp; by some&nbsp; HPV types,&nbsp; Gardasil,&nbsp; marketed&nbsp; by Merck&nbsp; and Cervarix,&nbsp; marketed&nbsp; by GlaxoSmithKline&nbsp; (Moscicki&nbsp; 2008). Both protect&nbsp; against initial infection&nbsp; with&nbsp; HPV types 16 and 18, which cause most of the HPV&nbsp; associated&nbsp; cancers.&nbsp; Gardasil also protects&nbsp; against HPV&nbsp; types&nbsp; 6 and 11, which cause 90% of genital warts (Asif et al. 2006; Kahn 2009; WHO 2009 &amp; Schil- ler et al. 2010). The vaccines provide little benefit to women who have already been infected with HPV types 16 and 18, which include most sexually active females. The World&nbsp; Health&nbsp; Organization (WHO&nbsp; 2009) has&nbsp; clear&nbsp; guidelines on HPV&nbsp; vaccination which&nbsp; outlines appropriate,&nbsp; cost-effective strategies for using HPV vaccines in public sector programs.&nbsp;&nbsp;</p>\r\n<p>\r\n	Cervical cancer is caused by a virus; it can be prevented in two ways: (i) by preventing&nbsp; HPV&nbsp; infection&nbsp; by&nbsp; using&nbsp; a poten- tial&nbsp; vaccine&nbsp; and&nbsp; (ii)&nbsp; by&nbsp; screening&nbsp; for&nbsp; the presence&nbsp; of&nbsp; cervical&nbsp; HPV&nbsp; infection&nbsp; and managing&nbsp; abnormal&nbsp; cytologies&nbsp; or&nbsp; pre-cancerous&nbsp; lesions&nbsp; before&nbsp; cancer&nbsp; develop.&nbsp; In Malaysia&nbsp; however,&nbsp; the&nbsp; lack&nbsp; of an&nbsp; adequate&nbsp; screening&nbsp; programme&nbsp; and awareness are the major factors contrib- uting&nbsp; to the&nbsp; high&nbsp; incidence&nbsp; of&nbsp; cervical cancer.&nbsp; The&nbsp; main&nbsp; purpose&nbsp; of&nbsp; this&nbsp; study was to establish a simple and rapid technique&nbsp; to&nbsp; detect&nbsp; high&nbsp; risk&nbsp; HPV&nbsp; subtypes using&nbsp; SyBrGreen&nbsp; Real-Time&nbsp; PCR.&nbsp; This technique is advantageous since it eliminates&nbsp; the&nbsp; need&nbsp; for&nbsp; gel&nbsp; electrophoresis and&nbsp; ethidium&nbsp; bromide&nbsp; staining.&nbsp; The&nbsp; method&nbsp; of&nbsp; SyBr-Green&nbsp; Real-Time&nbsp; PCR&nbsp; can be used as a screening approach to controlling cervical cancer.&nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS </strong></p>\r\n<p>\r\n	<em>Samples&nbsp;</em></p>\r\n<p>\r\n	Genomic&nbsp; DNA&nbsp; from&nbsp; 57&nbsp; HPV&nbsp; positive samples&nbsp; for&nbsp; HPV&nbsp; 16&nbsp; and&nbsp; HPV&nbsp; 18&nbsp; identified&nbsp; from&nbsp; our&nbsp; Type&nbsp; Specific-PCR&nbsp; (TS- PCR)&nbsp; was used for this study. The sam- ples&nbsp; include 37 CIN I, 12&nbsp; CIN II, 15 CIN III&nbsp; and&nbsp; 3&nbsp; squamous&nbsp; cell&nbsp; carcinoma (SCC).&nbsp; CaSki&nbsp; and&nbsp; HeLa&nbsp; cell&nbsp; lines&nbsp; were used as positive controls for HPV 16 and HPV&nbsp; 18,&nbsp; respectively.&nbsp; For&nbsp; negative&nbsp; control, blood DNA from normal women was chosen.&nbsp; DNA&nbsp; extraction&nbsp; was&nbsp; done&nbsp; using the&nbsp; QIAamp&nbsp; Tissue&nbsp; kit&nbsp; from&nbsp; QIAGEN (Germany).&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<em>Cell culture&nbsp;</em></p>\r\n<p>\r\n	The&nbsp; vial&nbsp; containing&nbsp; cell-lines&nbsp; was&nbsp; removed&nbsp; from&nbsp; the&nbsp; liquid&nbsp; nitrogen&nbsp; tank&nbsp; and was&nbsp; immediately&nbsp; thawed&nbsp; into&nbsp; a&nbsp; water bath at 37&ordm;C. The content of the vial was transferred into a centrifuge tube and 10 ml&nbsp; of&nbsp; growth&nbsp; medium&nbsp; were&nbsp; added.&nbsp; The tube&nbsp; was centrifuged&nbsp; at 1000 rpm for 10 minutes.&nbsp; Supernatant&nbsp; was&nbsp; removed&nbsp; and cells were resuspended in 5 ml of growth medium,&nbsp; later&nbsp; transferred&nbsp; to&nbsp; a&nbsp; culture flask and incubated in a 37&ordm;C CO2&nbsp; incubator. The medium was replaced the following&nbsp; day.&nbsp; Subculture&nbsp; was&nbsp; carried&nbsp; out by&nbsp; detaching&nbsp; cells&nbsp; using&nbsp; tripsin.&nbsp; One&nbsp; ml of trypsinethylenediaminetetraacetic acid (EDTA)&nbsp; solution&nbsp; (Flowlab)&nbsp; was&nbsp; added&nbsp; to the flask. The flasks were incubated in a CO2 incubator&nbsp; for&nbsp; 5&nbsp; minutes.&nbsp; The&nbsp; flask was&nbsp; then&nbsp; observed&nbsp; under&nbsp; the&nbsp; inverted microscope&nbsp; to&nbsp; check&nbsp; for&nbsp; the&nbsp; detachment of&nbsp; cells&nbsp; and&nbsp; was&nbsp; tapped&nbsp; gently&nbsp; to&nbsp; optimize cell detachment.&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<em>Real-Time PCR with SyBr Green&nbsp;</em></p>\r\n<p>\r\n	Genomic&nbsp; DNA&nbsp; from&nbsp; 57 positive&nbsp; samples for&nbsp; HPV&nbsp; 16 and&nbsp; HPV&nbsp; 18 were&nbsp; used&nbsp; to prepare&nbsp; a triplicate&nbsp; reaction&nbsp; for&nbsp; each sample&nbsp; using&nbsp; Real-Time&nbsp; PCR.&nbsp; Primers from&nbsp; TS-PCR&nbsp; (Walboomers&nbsp; et al.&nbsp; 1999) were used as a primer pair for HPV type 16&nbsp; and&nbsp; 18&nbsp; in&nbsp; the&nbsp; SyBr&nbsp; Green&nbsp; Real-Time PCR.&nbsp; Amplification&nbsp; reactions&nbsp; were&nbsp; per- formed&nbsp; with a volume&nbsp; of 12.5&micro;l&nbsp; of&nbsp; 2X SyBrGreen&nbsp; PCR&nbsp; Master&nbsp; Mix&nbsp; (Applied Biosystems), a total of 300nM of reverse and forward primers and 20ng of sample DNA in a 25&micro;l final volume. The protocol was set up at 95oC for 10 minutes to activate&nbsp; the Taq&nbsp; gold,&nbsp; 40 cycles&nbsp; at&nbsp; 95oC&nbsp; for 15&nbsp; sec&nbsp; and&nbsp; 60oC&nbsp; for&nbsp; 15 sec,&nbsp; consecu- tively.&nbsp; Each&nbsp; reaction&nbsp; was&nbsp; accompanied by a No Template Control (NTC) in which the template DNA was replaced by water. Amplification&nbsp; was&nbsp; performed&nbsp; in&nbsp; a 248- well PCR plate covered with optical caps in&nbsp; the ABI&nbsp; 7700HT-SDS&nbsp; real-time&nbsp; instrument (Applied Biosystem).</p>\r\n<p>\r\n	<strong>RESULTS </strong></p>\r\n<p>\r\n	<em>Optimization of SyBr Green Reaction&nbsp;</em></p>\r\n<p>\r\n	From our results using SyBr Green Real Time PCR, NTC was used as a negative control; CaSki and HeLa DNA were used as a positive control for HPV 16 and HPV 18&nbsp; respectively.&nbsp; Dissociation&nbsp; curve&nbsp; was generated&nbsp; to&nbsp; show&nbsp; the&nbsp; absence&nbsp; of&nbsp; non- specific&nbsp; amplification.&nbsp; The&nbsp; dissociation curve&nbsp; showed&nbsp; only&nbsp; a&nbsp; single&nbsp; peak&nbsp; at higher&nbsp; temperature&nbsp; and&nbsp; two&nbsp; peaks&nbsp; if&nbsp; primer-dimer&nbsp; occurred.&nbsp; According&nbsp; to&nbsp; Lamarcq et al. (2002), Cycle threshold (Ct) value&nbsp; was&nbsp; considered&nbsp; significant&nbsp; when&nbsp; it was&nbsp; at&nbsp; least&nbsp; four&nbsp; cycles&nbsp; lower&nbsp; than&nbsp; Ct value of the corresponding NTC. Another study by Szuhai et al. (2001), reported that a Ct value is significant when it was lower than 40 cycle&rsquo;s number. Figure (1a) shows the amplification plot of HPV 16 with negative control NTC, Ct value at 20 cycles. Primer-dimer of NTC was observed with a Ct value at 25 cycles. The Ct value of HPV 16 was significant because the differences between HPV 16 and NTC were five cycles. Figure (1b) shows dissociation curve of HPV 16. Two peaks were observed at 80.7&ordm;C for melt-ing temperature Tm of specific HPV 16 and at 74&ordm;C for a NTC. Figure (2a) shows the amplification plot of HPV 18 with NTC and Ct value for HPV 18 was 21 cycles. Primer-dimer of NTC was ob-served with a Ct value at 28 cycles. The Ct value of HPV 18 was significant because the differences between HPV 18 and NTC were seven cycles. Figure (2b) shows dissociation curve of HPV 18. Two peaks were observed at 79.3&ordm;C for melting temperature T<sub>m </sub> of specific HPV 18 and at 70&ordm;C for a NTC.</p>\r\n<p>\r\n	Identification of multiple infections of HPV types</p>\r\n<p>\r\n	Results from our previous TS-PCR, showed six samples were positive for multiple infections by HPV 16 and HPV 18. Figure 3 showed a dissociation curve of HPV 16 and HPV 18. Four peaks were displayed; one peak for HPV 16, one peak for HPV 18 and two peaks for NTC. Melting temperature T<sub>m </sub> for HPV 16 was 80.7&deg;C and for NTC was 74&deg;C, whereas for HPV 18, melting temperature Tm was 79.3&deg;C and for NTC was 70&deg;C. SyBr Green Real Time PCR method showed the Tm for HPV 16 and HPV 18 were different although the PCR products using TS-PCR for HPV 16 and HPV 18 produced a single band as a 100 bp. The dissociation curves using SyBr Green Real Time PCR method showed that the T<sub>m </sub> for HPV 16 was 80.6&deg;C whereas Tm for HPV 18 was 79.4&deg;C. Studies by Szu-hai et al. (2001) and Kleinle et al. (2002) suggested the T<sub>m </sub> of an amplification product is based on amplicon size and GC content; fragments of different length and G+C content can be distinguished by melting curve analysis. Our study found that the T<sub>m&nbsp; </sub>of HPV 16 was higher than T<sub>m </sub> of HPV 18.</p>\r\n<p>\r\n	<em>Identification of multiple infections of HPV types </em></p>\r\n<p>\r\n	Results from our previous TS-PCR, showed six samples were positive for multiple infections by HPV 16 and HPV 18. Figure 3 showed a dissociation curve of HPV 16 and HPV 18. Four peaks were displayed; one peak for HPV 16, one peak for HPV 18 and two peaks for NTC. Melting temperature T<sub>m </sub>for HPV 16 was 80.7&deg;C and for NTC was 74&deg;C, whereas for HPV 18, melting temperature T<sub>m </sub>was 79.3&deg;C and for NTC was 70&deg;C. SyBr Green Real Time PCR method showed the T<sub>m </sub>for HPV 16 and HPV 18 were different although the PCR products using TS-PCR for HPV 16 and HPV 18 produced a single band as a 100 bp. The dissociation curves using SyBr Green Real Time PCR method showed that the T<sub>m </sub>for HPV 16 was 80.6&deg;C whereas T<sub>m </sub>for HPV 18 was 79.4&deg;C. Studies by Szu-hai et al. (2001) and Kleinle et al. (2002) suggested the T<sub>m </sub>of an amplification product is based on amplicon size and GC content; fragments of different length and G+C content can be distinguished by melting curve analysis. Our study found that the T<sub>m </sub>of HPV 16 was higher than T<sub>m&nbsp; &nbsp;</sub>of HPV 18.</p>\r\n<p>\r\n	Table 1 showed the C<sub>t</sub>&nbsp;value of HPV 16 and HPV 18 were below 40 cycles and also differs from NTC by six cycles so that the C<sub>t </sub>values were significant based on studies by Szuhai et al. (2001) and Lamarcq et al. (2002). This table also showed the differences of T<sub>m </sub>between specific product of HPV 16 and NTC were 6.7&deg;C whereas the differences of T<sub>m </sub>between specific product of HPV 18 and NTC were 9.3&deg;C. Study by Kleinle et al. (2002), suggested the T<sub>m</sub>of the gene specific product differs from primer-dimers by at least 10&deg;C, the peak of the gene-specific product did not overlap with the primer-dimer peak and thus the accuracy of the quantification was validated.</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	<em>Amplification plot of HPV 16 and HPV 18 </em></p>\r\n<p>\r\n	In this study, minimum primers for HPV 16 and 18 were 300:300nM for forward and reverse primers and DNA concentration at 100 &eta;g. All graphs from Figure 1-3 showed C <sub>t </sub>value for HPV 16 was 20&plusmn;1 cycles and dissociation curve of HPV 16 with melting curve T<sub>m </sub>was 80.7&plusmn;0.2&ordm;C whereas melting curve T<sub>m </sub>for NTC was 73.5&plusmn;0.2&ordm;C. Amplification plot of HPV 18 showed presence of two curves with one specific for HPV 18 and another one for NTC. C<sub>t </sub>value for HPV 18 was 21&plusmn;1cycles and C<sub>t </sub>value for NTC was 28&plusmn;1 cycles. Table 1 showed the C<sub>t </sub>value of HPV 16 and 18 were significant because, the difference between specific product and NTC was five and seven cycles (Kleinle 2002). For melting temperature, T<sub>m </sub>shows the differences of T<sub>m </sub>between HPV 16 and NTC were below 10&deg;C such as 7.2&deg;C whereas for HPV 18 the differences between specific HPV 18 and NTC were 9.4&deg;C. Kleinle et al. (2002), suggested the T<sub>m </sub>of the gene specific product differs from primer-dimers or NTC by at least 10&deg;C, the peak of the gene-specific product did not overlap with the primer-dimer peak and thus the accuracy of the quantification was validated. However, this study found the differences of T<sub>m </sub>for HPV 16 and HPV 18 were below 10&deg;C, this phenomena happened because the amplicon size of HPV 16 and 18 were 100bp and the size of NTC range from 50-60bp. The close-up of amplicon size between specific product and NTC will affect the T<sub>m</sub>. Although the amplicon size between spe-cific products and NTC was very close, the graph shows gene-specific product of HPV 16 and HPV 18 did not overlap with the NTC peak so the accuracy of the qualification was validated.</p>\r\n<p>\r\n	<em>Identification of multiple infections of HPV types </em></p>\r\n<p>\r\n	From our previous sequencing results of PCR products of E7 gene for HPV 16 (RID 1055387141-08795-2060) and HPV 18 (RID 1056435350-019738-16228) we observed that, GC content for HPV 16 was higher than the GC content of HPV 18, and the PCR product of HPV 16 had high T<sub>m </sub>compared to T<sub>m </sub>of HPV 18. The differences in melting temperature can be used as a method to identify and diffe-rentiate multiple infections between HPV16 and HPV 18. Kleinle et al. (2002) and Lamarcq et al. (2002) reported that multiple specific products can be sepa-rated from each other due to different T<sub>m </sub>in most cases. This method is advantageous since it eliminates the need for gel electrophoresis. This finding shows, SyBr Green can be used to differentiate and identify HPV types. Table 1 shows a summary of the C<sub>t </sub>values and T<sub>m </sub>for HPV 16 and HPV 18. The C<sub>t </sub>value of HPV 16 and HPV 18 were below 40 cycles and also differ from NTC by six cycles, indicating that the C<sub>t </sub>values were significant. The findings were supported by studies by Szuhai et al. (2001) and Lamarcq et al. (2002). Table 1 also showed the differences of T<sub>m</sub>between HPV 16 and HPV 18 with the NTCs being 6.7&deg;C and 9.3&deg;C respectively. The peak of the gene-specific product did not overlap with the primer-dimer peak and thus the accuracy of the quantification was validated. This SyBr Green Real- Time PCR method offers the possibility of substantially increasing the throughput of sample analysis, of analyzing different samples for the presence of any of these gene mutations and/or determining the individual gene mutation pattern. In terms of the rapidity, flexibility and the subsequent economic advantages, this approach is suitable for use in clinical and routine laboratory applications (Cubie et al. 2001; Szuhai et al. 2001; Kleinle et al. 2002; Villa et al. 2006).</p>\r\n<p>\r\n	<strong>CONCLUSION </strong></p>\r\n<p>\r\n	Using SyBr Green Real-Time PCR technique, minimum forward and reverse primers for each HPV type 16 and 18 were required. DNA concentration for each samples were fixed at 100ng. SyBr Green Real-Time PCR method has the advantage of it being able to be used directly to detect and identify the HPV types based on amplification plot and melting temperature graphs. This SyBr Green Real-Time PCR method has the potential to be used as a method for de-tection and identification of HPV types in CINs and cervical cancer patients in Malaysia. In addition it has the advantage of detecting multiple infections of HPVs which adds to its economic value.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS</strong></p>\r\n<p>\r\n	The study was supported by a postgraduate grant from Institute Bioscience (IRPA-5495304), Universiti Putra Malaysia. The authors wish to thank the Director General of Health, Malaysia for permission to publish this scientific paper. We also like to thank the Deputy Director General (Research &amp; Technical Support, MOH) and the Director of IMR for their support.</p>\r\n', 2);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(464, 461, '<p>\r\n	<strong>INTRODUCTION </strong></p>\r\n<p>\r\n	One of the important objectives of quality assurance for a laboratory is to deliver accurate, timely, and clinically relevant diagnostic reports to the customers i.e. the clinicians and patients. The quality of this service is thus related, among other factors, to the accurateness and timeli-ness of the reports. For a pathology re-port to be beneficial to the clinicians, re-sults should be available on time espe-cially for urgent requests from critical units.</p>\r\n<p>\r\n	The turn-around time (TAT) can be de-fined as the time taken from when the specimen arrives at the laboratory to the time results are released. It is one of the indicators of quality and efficacy, princi-pally in laboratories performing urgent analyses (Hilborne et al. 1989). This is of importance especially when processing samples for coagulation tests where standard processing methods may not be fast enough for urgent requests.</p>\r\n<p>\r\n	A survey in the United States showed that the prothrombin time (PT) and acti-vated partial thromboplastin time (APTT) tests represent more than 90% of co-agulation tests ordered (Shahangian et al. 2005) which mirrors the requested tests in our laboratory. In coagulation testing, platelet poor plasma (PPP) is required to prevent interference by the phospholipid surface on the platelets. With standard laboratory methods (Ad-cock et al. 2008), the duration for reporting coagulation tests is about 40&ndash;180 minutes. A long TAT may cause adver-sity in medical situations associated with critical, surgical and emergency cases, and good medical care could be denied (Steindel &amp; Howanitz 2001).</p>\r\n<p>\r\n	Strategies to reduce the time essential for specimen processing without affecting quality should be recognized, especially for laboratories performing urgent ana-lyses. In our laboratory, urgent cases are mainly requested by the Emergency Department. The standard methods for processing coagulation tests do not meet the current TAT for urgent requests (30 minutes) in our centre. This study was performed to determine whether a higher centrifugation speed and a shorter centrifugation time would affect coagulation results as compared to centrifugation using the standard time (10-15 minutes) for PPP preparation (Ad-cock et al. 2008).</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS </strong></p>\r\n<p>\r\n	Blood samples for routine coagulation test were randomly selected from patients from November through December 2010. The age range for the study population was from 28 to 83-years-old.</p>\r\n<p>\r\n	Platelet-poor plasma (PPP) was pre-pared at room temperature within one hour of sample collection using two table- top centrifuge machines (Rotina 38, Hettich Zentrifugen, Germany). Each sample was thoroughly mixed and separated into equal volumes in two separate plastic vacuum tubes (with 3.2% sodium citrate). The first tube was centrifuged at 4000 rpm for five minutes (protocol A), while the second tube was centrifuged at 3580 rpm for 15 minutes (protocol B) simulta-neously. A plastic pipette was used to carefully transfer approximately 1ml of supernatant plasma from the middle of the sample away from the buffy coat to avoid contamination with excess platelets.</p>\r\n<p>\r\n	All paired samples were prepared and tested for PT, APTT and fibrinogen at room temperature immediately after cen-trifugation using a coagulation analyzer with the required controls and reagents (STA-Compact, All-Eights, France). The PPP samples were tested for platelet count within 30 minutes of collection us-ing a haematology analyser (LH 750, Beckmann Coulter, USA). Data were entered using Microsoft Ex-cel and descriptive statistical analyses (mean, range, standard deviation (SD) and paired Student <em>t</em>-test) were computed using SPSS, version 14.</p>\r\n<p>\r\n	<strong>RESULTS </strong></p>\r\n<p>\r\n	A total of 79 samples were randomly selected for this study. Of these 79 samples, 49 samples showed abnormal PT, 30 samples showed normal PT, 34 samples showed abnormal APTT and 44 samples showed normal APTT readings. One APTT reading (21.5s) was much lower than the normal reference range and was thus excluded from the data analysis. The normal reference range for PT and APTT are 11.4-14.2 seconds and 31.3-46.1 seconds respectively. Platelet counts less than 10x10<sup>9</sup>/L were produced from all 79 PPP samples centrifuged for 15 minutes at 3580 rpm ex-cept for two samples (2.5%) which showed a platelet count of more than 10x10<sup>9</sup>/L (12x10<sup>9</sup>/L and 14x10<sup>9</sup>/L). However, when samples were centrifuged for five minutes at 4000 rpm, only 15 sam-ples (19%) showed platelet counts less than 10x10<sup>9</sup>/L. The difference between these two groups were statistically significant (p&lt;0.001).</p>\r\n<p>\r\n	Descriptive statistics for platelet count, PT, APTT and fibrinogen levels prepared for five minutes at 4000 rpm (protocol A) and 15 minutes at 3850 rpm (protocol B) are shown in Table 1. We found a significant difference (p=0.01) between the normal PT levels when comparing centrifugation between 15 minutes (standard time) and five minutes. However, there was no significant difference in the results of normal APTT, abnormal PT and APTT between the two protocols. There was also good correlation between the results of PT, APTT and fibrinogen levels of protocol A and B respectively (Figure 1, 2 and 3).</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	Preparation of blood specimens can be a major time limiting factor in the laboratory throughput. In order to obtain a faster TAT, while maintaining sample quality and accuracy of results, the time con-sumed on coagulation sample collection, handling, processing and testing should be looked into (Lawrence 2003). This is especially so in critical areas where results are needed urgently.</p>\r\n<p>\r\n	In our laboratory, it takes about 60 minutes to release routine coagulation results to the wards or clinics. The time may be further prolonged if repeat testing is necessary. This will not fulfill the TAT requirement for urgent tests for critical units such as the Emergency Department and Intensive Care Unit. The shortest recommended centrifugation time to produce PPP with platelet counts less than 10x10<sup>9</sup>/L is 10-15 minutes at 3850 rpm using regular centrifuges (Adcock et al. 2008). In this study, we looked at whether centrifugation at five minutes would affect coagulation results (PT/APTT), as compared to centrifugation at 15 minutes recommended time.</p>\r\n<p>\r\n	We found that 15-minutes centrifugation, achieved a better PPP than a five minutes centrifugation. A shorter centrifugation time (five minutes) causes an increase in platelet count in the sample tubes in 81% of cases. Our results dif-fered from a previous report by Sultan (2010) on 46 healthy volunteers which showed nearly all samples centrifuged at five minutes had platelet count less than 10x109. Barnes and colleagues (2002) showed that 10 minutes was the minimum centrifugation time required to consistently meet the recommended standards.</p>\r\n<p>\r\n	However, although platelet counts were significantly different in both protocols, the coagulation results between 15 minutes recommended centrifugation time and five minutes centrifugation time did not differ significantly for abnormal PT, normal APTT, abnormal APTT and fibrino-gen results (Table 1). This is in agreement with a previous study (Barnes et al. 2002). For normal PT results, the difference between 15 minutes centrifugation and five minutes centrifugation had a p value of 0.01. Nevertheless, the mean (13.1s vs 13.3s) and SD (0.58 vs 0.57) did not differ much. In our opinion, this unexpected result may be due to statistical reason as a result of a small number of samples for this subgroup, rather than the platelet content in the PPP itself. APTT is more sensitive to platelet con-tamination than PT. Therefore platelet contamination will affect the APTT result more than the PT result, a finding of which was not evident in this study.</p>\r\n<p>\r\n	When we combined both normal and abnormal PT and APTT results, we observe a good correlation between the two different protocols used in this study (Figure 1 and 2). This is also true for fibrinogen levels (Figure 3). In view of this, we feel that although the platelet count can be high when a sample is centrifuged for five minutes, it does not affect coagulation results in general.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	This study showed that, although there was presence of residual platelet counts in PPP prepared with five minutes cen-trifugation at 4000 rpm, no significant difference was found for the results of PT, APTT and fibrinogen values. We conclude that this centrifugation time can be used for urgent test requests without compromising the quality and reliability of the results. This contributes to reducing the overall turnaround time for coagula-tion STAT results and thus provides bet-ter medical care for patients with coagulation test requests.</p>\r\n', 1),
(465, 462, '<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Children with Down syndrome have four to five times higher risk of developing renal and urinary tract anomalies as compared to the general population (Kupferman et al. 2009). These anomalies include renal hypoplasia, hydrourete-ronephrosis, ureterovesical and uropelvic junction obstruction, vesicoureteric reflux (VUR) and posterior urethral valves (PUV). Of these, PUV is the least common anomaly (Narasimhan et al. 2005).</p>\r\n<p>\r\n	Usually, PUV is detected during the prenatal or neonatal period. However, delay in presentation and diagnosis of PUV in particular in Down syndrome (DS) has been reported recently (Kupferman et al. 2009 &amp; Culty et al. 2007). This is contributed by lack of awareness of the association between PUV and DS (Narasimhan et al. 2005). With this in mind, we present a child with Down syndrome who had recurrent orchitis as the initial presentation of a posterior urethral valve that had a delayed diagnosis.</p>\r\n<p>\r\n	With this in mind, we present a child with Down syndrome who had recurrent orchitis as the initial presentation of a posterior urethral valve that had a delayed diagnosis.</p>\r\n<p>\r\n	<strong>CASE REPORT </strong></p>\r\n<p>\r\n	CKS is 5-year-old Chinese boy who was delivered via Caesarean section at 37 weeks period of gestation (POG) with a birth weight of 2.87kg. His mother was a 32-year-old lady gravida 2 para 0+1 (with one history of abortion). She had gesta-tional diabetes mellitus and was referred to our institution at 26 weeks POG. She was started on subcutaneous insulin in-jection for her diabetic treatment at 35 weeks POG. Fetal echocardiography re-vealed a fetal cardiac anomaly. An amni-ocentesis and karyotyping was done at 35 weeks POG that revealed a male fetus with trisomy 21.</p>\r\n<p>\r\n	Postnatally, he was admitted to the neo-natal intensive care unit (NICU) for car-diac evaluation and treatment because he had signs and symptoms of cardiac failure with scalp oedema and bilateral pedal oedema. An echocardiography was done and revealed a large ventricular septal defect (VSD), atrial septal defect (ASD) and a small patent ductus arteriosus (PDA). He was treated for the cardiac fail-ure during his stay in the NICU. He un-derwent corrective cardiac surgery at the age of 5 months old.</p>\r\n<p>\r\n	He was diagnosed to have left orchitis at the age of one year when he presented with a left inguinal pain and swelling. An ultrasound of the renal tract and scrotum revealed bilateral undescended testes at the inguinal region with evidence of increased colour Doppler flow of the left testis indicating orchitis (Figure 1). Both of his kidneys were normal. His urine FEME showed presence of leucocytes but the urine culture was negative. His renal pro-file was normal. His condition improved with a course of antibiotics. A micturating cystourethrography (MCU) was planned but the patient did not turn up for the ap-pointment. Later, he had another episode of orchitis at the age of 5 when he presented with left inguinal swelling. There was no history of difficult urination or urinary dribbling.</p>\r\n<p>\r\n	Clinically, he was afebrile. His left inguinal region was swollen and tender. Bio-chemical investigations showed normal renal profile and urine culture. An ultra-sound showed similar findings as his previous presentation. Micturating cystourethrography was requested inview of the recurrent orchitis, which revealed a posterior urethral valve. There was no vesi-coureteric reflux (Figure 2).</p>\r\n<p>\r\n	The patient subsequently underwent cystoscopy. Intraoperative findings showed a type I posterior urethral valve associated with glanular hypospadias. The bladder neck, bladder outline and both ureteric orifices were normal. The PUV was fulgurated at 4 and 8 o&rsquo;clock position with a resectoscope. The child was well post-operatively. DISCUSSION Down syndrome (DS) is a common chromosomal disorder. There is an in-creased risk of congenital abnormalities, including cardiac, eye, and gastrointestinal defects. Genitourinary tract abnormalities however, are not often high-lighted (Kupferman et al. 2009; Narasim-han et al. 2005). Kupferman et al. (2009) reported that the prevalence of renal and urinary tract anomalies in children with Down syndrome was 3.2% as compared to the general population, which is 0.7%. This included, in descending frequency: hydronephrosis, hydroureter, hypospadias, renal agenesis, cystic dysplastic kidney, posterior urethral valve, anterior urethral obstruction and prune belly syndrome. There were few cases of PUV in DS reported, which presented at a variable range of age (Kupferman et al. 2009 &amp; Narasimhan et al. 2005). A retrospective study done from year 1992-2004 re-vealed that there were only two PUV cases amongst 123 patients with Down syndrome with underlying renal and urinary tract anomalies (Kupferman et al. 2009). On the other hand, Narasimhan et al. (2005) reported that six out of 230 patients with PUV had Down syndrome. Their age of presentation ranged be-tween 18 months to seven years. He concluded that in view of the mental re-tardation and other social problems, families of patients with Down syndrome might not report the poor urinary stream, which contributes to the delay in the diagnosis. The diagnosis of PUV in this patient was delayed, as there was no subsequent follow-up in the clinic. Therefore, further investigations of the epididymo-orchitis (EO) was not done.</p>\r\n<p>\r\n	There are many different clinical presentations of PUV. Most patients presented with urinary incontinence/ dribbling, obstructive uropathy and urinary tract infection (Kupferman et al. 2009; Narasimhan et al. 2005 &amp; Culty et al. 2007). Khalid et al. (2001) reported that the presentation of PUV is variable whe-reby 27% of patients were diagnosed by antenatal ultrasound, 30.4% presented with urinary tract infection, 32.4% of patients had history of poor urinary stream, 7.4% of patients had retention of urine in neonatal life and 2.8% had symptoms of renal impairment. Our patient was diagnosed as PUV after he presented with recurrent EO. Surprisingly, he did not have any symptoms of bladder outlet obstruction. A study done among non-DS children with PUV showed that 7.1% of cases presented with recurrent urinary tract infection had history of recurrent EO (Ikuerowo et al. 2008). Abbas et al. (2008) reported a case of PUV in a 4-year-old non-DS child who presented with right scrotal swelling and was diag-nosed to have EO. There was no docu-mented symptoms of bladder outlet ob-struction. He had a history of urinary tract infection at the age of three years old. We postulate that the recurrent left EO was likely due to reflux of urine through the ejaculatory duct and vas deferens into the epididymis and testis, which caused chemical irritation.</p>\r\n<p>\r\n	Generally, PUV was diagnosed early antenatally, during newborn or infancy (Belman et al. 2002). Antenatally, PUV can be diagnosed by identifying typical &lsquo;key-hole&rsquo; appearance of the distended bladder and the dilated posterior urethra. Postnatally, ultrasound scan demonstrates dilated posterior urethra, bilateral hydronephrosis and/ hydroureters, thick-walled urinary bladder and bladder diver-ticuli. Micturating cystourethrography (MCU) will further delineate the anatomy of the urethra and shows transverse filling defect in the posterior urethra, which represents the membrane of the PUV (Belman et al. 2002; Ikuerowo et al. 2008). Traditional teaching suggests EO is rare in children. It has been recommended that all boys with EO should un-dergo investigation of the urinary tract. The investigation varies with some cen-ters, which include renal tract ultrasound, cystograms, cystoscopy, and/or urodynamics (McAndrew et al. 2002).</p>\r\n<p>\r\n	Posterior urethral valve obstruction is a significant cause of renal-related morbidity in male children. Obstructive uropathy from congenital obstructive posterior urethral membrane results in end stage renal disease in 30-40% of patients and is a common indication for renal dialysis and transplantation in patients less than 5 years of age (Ikuerowo et al. 2008). Fortunately, this complication did not present in our patient. He was lucky since he has no vesicoureteric reflux which is one of the favourable prognostic factor in PUV (Belman et al. 2002). A study done by Kukreja et al. (2001) reported that 29% of 70 PUV patients had renal insufficiency at the end of a 3-year follow-up. He found out that factors found to be statistically significant were age at intervention more than two years, recurrent urosepsis, bilateral high grade vesico-ureteric reflex, bilateral paren-chymal damage as seen on ultrasonography and nadir serum creatinine of more than 0.8 mg% (p value &lt;0.05). Therefore, this patient has to be closely monitored to asses progression of his renal function in order to prevent renal insufficiency later.</p>\r\n<p>\r\n	<strong>CONCLUSION</strong></p>\r\n<p>\r\n	It is important to diagnose PUV early especially in DS patients as the obstructive symptoms may not be evident at clinical presentation. With the association be-tween DS and PUV as well as other renal and urinary tract anomalies, patients with Down syndrome should be screened by doing ultrasound of the renal tract followed by micturating cystourethrogram (MCU) if abnormal.</p>\r\n', 1),
(466, 463, '<p class="rtejustify">\r\n	&nbsp;INTRODUCTION&nbsp;&nbsp;</p>\r\n<p class="rtejustify">\r\n	Lifestyle&nbsp; factors&nbsp; are&nbsp; associated&nbsp; with chronic&nbsp; diseases&nbsp; such&nbsp; as&nbsp; hypertension, coronary&nbsp; heart&nbsp; disease,&nbsp; cancer&nbsp; and diabetes mellitus (DM). Globally, the pre- valence of DM is on the increasing trend (Wild&nbsp; et&nbsp; al.&nbsp; 2004).&nbsp; In&nbsp; Malaysia,&nbsp; the&nbsp; third National Health and Morbidity Surveys in 2006&nbsp; showed&nbsp; that&nbsp; the&nbsp; prevalence&nbsp; of&nbsp; DM for&nbsp; those&nbsp; aged&nbsp; 30&nbsp; years&nbsp; and&nbsp; above&nbsp; was 14.9%&nbsp; compared&nbsp; to&nbsp; 8.3%&nbsp; in&nbsp; a&nbsp; similar survey in 1996.</p>\r\n<p class="rtejustify">\r\n	DM&nbsp; is&nbsp; more&nbsp; prevalent&nbsp; among&nbsp; the el- derly,&nbsp; as there&nbsp; is an&nbsp; increased&nbsp; in elderly population.&nbsp; In Malaysia,&nbsp; 5.2%&nbsp; of&nbsp; the populations were elderly in 1970, 6.3% in 2000&nbsp; and&nbsp; projected&nbsp; to 10.0%&nbsp; in&nbsp; 2020 (Population&nbsp; Ageing&nbsp; Trends&nbsp; in Malaysia 2005).&nbsp;&nbsp;</p>\r\n<p class="rtejustify">\r\n	DM&nbsp; is&nbsp; more common&nbsp; in the elderly&nbsp; be- cause&nbsp; as&nbsp; a person&nbsp; grows&nbsp; older,&nbsp; the&nbsp; de- position&nbsp; of&nbsp; body&nbsp; fat&nbsp; increases.&nbsp; For&nbsp; those aged&nbsp; above&nbsp; 30&nbsp; years,&nbsp; postprandial&nbsp; glu- cose will increase five mg/dL for every 10 years&nbsp; of&nbsp; age. The&nbsp; rate&nbsp; of&nbsp; glucose&nbsp; ab- sorption&nbsp; and insulin&nbsp; production&nbsp; becomes slower&nbsp; after&nbsp; meals.&nbsp; Insulin&nbsp; resistance increases&nbsp; because&nbsp; of&nbsp; reduction&nbsp; in body mass&nbsp; and increase&nbsp; in body&nbsp; fat&nbsp; (Hall&nbsp; &amp; Zweig 2005).</p>\r\n<p class="rtejustify">\r\n	Glucose&nbsp; control&nbsp; is&nbsp; important,&nbsp; as&nbsp; hyper- glycaemia&nbsp; is&nbsp; one&nbsp; of&nbsp; the&nbsp; risk&nbsp; factors&nbsp; for the&nbsp; development&nbsp; of&nbsp; DM&nbsp; complications. For those who are 60 to 69 years old, the life expectancy will be reduced by four to five&nbsp; years,&nbsp; and&nbsp; for&nbsp; those&nbsp; who&nbsp; are&nbsp; more than 70 years old, type 2 DM reduces life expectancy by three years (Hall &amp; Zweig 2005).&nbsp; Microvascular&nbsp; diseases&nbsp; are&nbsp; the main cause of blindness and 50% of the amputation&nbsp; cases&nbsp; not&nbsp; caused&nbsp; by&nbsp; injuries or&nbsp; accidents&nbsp; are&nbsp; due to DM.&nbsp; Twenty&nbsp; five percent&nbsp; of&nbsp; new&nbsp; cases&nbsp; of&nbsp; haemodialysis are caused by DM. The risk of ischaemic heart&nbsp; diseases&nbsp; and&nbsp; stroke&nbsp; increased&nbsp; by two&nbsp; to&nbsp; four&nbsp; times.&nbsp; Nearly&nbsp; 80%&nbsp; of&nbsp; elderly diabetics&nbsp; die due&nbsp; to heart&nbsp; diseases&nbsp; and other&nbsp; vascular&nbsp; diseases&nbsp; (Roos&nbsp; &amp;&nbsp; Samos 2002).</p>\r\n<p class="rtejustify">\r\n	However,&nbsp; there&nbsp; are&nbsp; multiple&nbsp; issues&nbsp; in the&nbsp; management&nbsp; of&nbsp; elderly&nbsp; diabetics&nbsp; in- cluding&nbsp; polypharmacy,&nbsp; lack&nbsp; of health knowledge and financial problems (Haas 2006).&nbsp; Furthermore&nbsp; the diabetes&nbsp; popula- tion is also associated with geriatric syn- drome&nbsp; such&nbsp; as&nbsp; functional&nbsp; disabilities, depression,&nbsp; fall,&nbsp; urinary&nbsp; incontinence, pain&nbsp; and&nbsp; dementia,&nbsp; which&nbsp; occur&nbsp; due to the&nbsp; ageing&nbsp; and&nbsp; diabetic&nbsp; complications. These&nbsp; often&nbsp; lead to frailty,&nbsp; loss&nbsp; of&nbsp; inde- pendence and low quality of life (Araki &amp; Ito 2009). Therefore, this group of people often&nbsp; needs&nbsp; help&nbsp; from&nbsp; their&nbsp; family members.</p>\r\n<p class="rtejustify">\r\n	Good&nbsp; glucose&nbsp; control&nbsp; requires&nbsp; the commitment and motivation of their fami- lies.&nbsp; It may&nbsp; reduce&nbsp; the&nbsp; risk&nbsp; of&nbsp; diabetes complications&nbsp; and&nbsp; ensures&nbsp; a better quality&nbsp; life&nbsp; for&nbsp; the elderly&nbsp; (Hall&nbsp; &amp; Zweig 2005). Lack of social support may lead to the&nbsp; aggravation&nbsp; of&nbsp; geriatric&nbsp; symptoms such as functional disability, malnutrition, depression&nbsp; and&nbsp; cognitive&nbsp; impairment among&nbsp; elderly&nbsp; diabetics&nbsp; (Araki&nbsp; &amp; Ito 2009). Higher level of social support has a role for better glucose control (Okura et al.&nbsp; 2009).&nbsp; Reblin&nbsp; &amp; Uchino&nbsp; (2008)&nbsp; found that&nbsp; social&nbsp; and&nbsp; emotional&nbsp; support&nbsp; from others can be protective for the persons&rsquo; health&nbsp; status.&nbsp; However,&nbsp; this&nbsp; finding&nbsp; was different&nbsp; from&nbsp; White&nbsp; et al.&nbsp; (2009)&nbsp; which showed&nbsp; that&nbsp; the elderly&nbsp; who&nbsp; needed more social support&nbsp; was associated&nbsp; with poorer health outcome.</p>\r\n<p class="rtejustify">\r\n	Glucose control is measured by glyco- sylated&nbsp; haemoglobin&nbsp; (HbA1c).&nbsp; For&nbsp; every 1%&nbsp; rise&nbsp; in&nbsp; HbA1c,&nbsp; there&nbsp; is&nbsp; a 35 mg/dL rise in blood glucose. Red blood cell&nbsp; will be&nbsp; replenished&nbsp; for&nbsp; every&nbsp; 8&nbsp; to 12&nbsp; weeks, making&nbsp; HbA1c&nbsp; the&nbsp; best&nbsp; method&nbsp; of&nbsp; mea- suring glucose control over the long term (Alam et al. 2005).</p>\r\n<p class="rtejustify">\r\n	&nbsp;The&nbsp; objective&nbsp; of&nbsp; this&nbsp; study&nbsp; was&nbsp; to&nbsp; de- termine the prevalence of glucose control among&nbsp; elderly&nbsp; diabetics&nbsp; and&nbsp; its&nbsp; asso- ciated&nbsp; factors.&nbsp; Factors&nbsp; studied&nbsp; were grouped&nbsp; into&nbsp; socio-demographic,&nbsp; history of&nbsp; diseases,&nbsp; diabetes&nbsp; knowledge&nbsp; and family&nbsp; support.&nbsp; In&nbsp; this&nbsp; study,&nbsp; HbA1c&nbsp; of 6.5%&nbsp; or&nbsp; less&nbsp; was&nbsp; considered&nbsp; as&nbsp; good glucose control (CPG DM 2004).&nbsp;</p>\r\n<p class="rtejustify">\r\n	&nbsp;MATERIALS AND METHODS</p>\r\n<p class="rtejustify">\r\n	&nbsp;This cross sectional study was conducted from February until May 2009 in Kulim. It involved&nbsp; 329&nbsp; elderly&nbsp; diabetics,&nbsp; chosen through&nbsp; stratified&nbsp; random&nbsp; sampling&nbsp; from seven government health clinics in Kulim. Respondents&nbsp; were chosen based on the proportion&nbsp; of&nbsp; the&nbsp; registered&nbsp; diabetic&nbsp; pa- tients in each clinic.&nbsp; The inclusion criteria were&nbsp; elderly&nbsp; diabetics&nbsp; aged&nbsp; 60&nbsp; and above,&nbsp; and&nbsp; who&nbsp; had&nbsp; been&nbsp; diagnosed with&nbsp; DM&nbsp; for&nbsp; at&nbsp; least&nbsp; one&nbsp; year.&nbsp; The exclusion&nbsp; criteria&nbsp; were&nbsp; aphasic respondents,&nbsp; patients&nbsp; who&nbsp; were&nbsp; on haemodialysis and dementia.&nbsp;</p>\r\n<p class="rtejustify">\r\n	Respondents&nbsp; were&nbsp; interviewed&nbsp; perso- nally by the researcher using a structured questionnaire.&nbsp; For&nbsp; diabetes&nbsp; knowledge assessment,&nbsp; the&nbsp; questions&nbsp; were&nbsp; based on&nbsp; the&nbsp; Diabetes&nbsp; Knowledge&nbsp; Test&nbsp; pre- pared&nbsp; by&nbsp; Diabetes&nbsp; and&nbsp; Hormone&nbsp; Center of&nbsp; the&nbsp; Pacific.&nbsp; Evaluation&nbsp; for&nbsp; family&nbsp; support&nbsp; was&nbsp; through&nbsp; a&nbsp; questionnaire adopted&nbsp; from&nbsp; the&nbsp; Duke-University&nbsp; of North Carolina Functional Social Support Questionnaire,&nbsp; with&nbsp; some&nbsp; modification made&nbsp; to&nbsp; suit&nbsp; the&nbsp; Malaysian&nbsp; community.&nbsp; The questionnaire was validated and the Cronbach&rsquo;s&nbsp; Coefficient&nbsp; Alpha&nbsp; for&nbsp; the scale of DM knowledge was 0.69 and for family&nbsp; support&nbsp; was&nbsp; 0.85.&nbsp; To&nbsp; determine the&nbsp; HbA1c&nbsp; level,&nbsp; two&nbsp; ml&nbsp; of&nbsp; blood&nbsp; was collected from each respondent.&nbsp;</p>\r\n<p class="rtejustify">\r\n	Statistical&nbsp; Package&nbsp; of&nbsp; Social&nbsp; Science (SPSS) software, version 12.0&nbsp; was used in&nbsp; processing&nbsp; and&nbsp; analyzing&nbsp; the&nbsp; data. The significant level at p &lt; 0.05 and 95% confidence interval (CI) were used in this study.&nbsp; The&nbsp; Research&nbsp; and&nbsp; Ethics&nbsp; Com- mittee,&nbsp; Universiti&nbsp; Kebangsaan&nbsp; Malaysia as well as the Ministry of Health (NMRR- 08-1194-1453)&nbsp; approved&nbsp; this&nbsp; study. Written&nbsp; and&nbsp; verbal&nbsp; consent&nbsp; were&nbsp; taken from each respondent.</p>\r\n<p class="rtejustify">\r\n	RESULTS&nbsp;</p>\r\n<p class="rtejustify">\r\n	The&nbsp; prevalence&nbsp; of&nbsp; good&nbsp; glucose&nbsp; control in&nbsp; this&nbsp; study&nbsp; was&nbsp; 22.5%.&nbsp; Three&nbsp; hundred and&nbsp; thirty&nbsp; one&nbsp; respondents&nbsp; were&nbsp; ap- proached&nbsp; and&nbsp; 329&nbsp; agreed&nbsp; to&nbsp; participate in&nbsp; this&nbsp; study.&nbsp; The&nbsp; response&nbsp; rate&nbsp; of&nbsp; this study&nbsp; was&nbsp; 99.4%.&nbsp; Women&nbsp; made&nbsp; up 52.9%&nbsp; of&nbsp; the&nbsp; respondents.&nbsp; Ethnicity&nbsp; dis- tributions&nbsp; were&nbsp; Malays&nbsp; 58.4%,&nbsp; Indians 28.6%, Chinese 12.2% and others 0.9%. The&nbsp; majority&nbsp; of&nbsp; the&nbsp; respondents&nbsp; was&nbsp; in the&nbsp; 60&nbsp; to&nbsp; 69&nbsp; years&nbsp; old&nbsp; age&nbsp; group (73.9%),&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; married&nbsp; (68.7%)&nbsp; and&nbsp; attained up to primary education (76.3%).&nbsp;&nbsp;</p>\r\n<p class="rtejustify">\r\n	The&nbsp; majority&nbsp; of&nbsp; the elderly&nbsp; were&nbsp; diag- nosed&nbsp; as&nbsp; DM&nbsp; for&nbsp; more&nbsp; than five&nbsp; years (68.1%).&nbsp; With&nbsp; regards&nbsp; to co-morbidities, 79.6%&nbsp; had&nbsp; hypertension,&nbsp; 46.5%&nbsp; dyslipi- daemia,&nbsp; 23.1%&nbsp; ischaemic&nbsp; heart&nbsp; diseases,&nbsp; 10.9%&nbsp; cataract&nbsp; and&nbsp; retinopathy, 9.1% joint&nbsp; and&nbsp; bone&nbsp; problems and 2.1% history&nbsp; of&nbsp; stroke.&nbsp; Those&nbsp; who&nbsp; had&nbsp; hypertension,&nbsp; dyslipidaemia,&nbsp; ischemic&nbsp; heart diseases&nbsp; and&nbsp; stroke&nbsp; were&nbsp; grouped&nbsp; as having&nbsp; co-morbidities.&nbsp; Overall,&nbsp; 85.7% had&nbsp; co-morbidities.&nbsp; Bivariate&nbsp; analysis showed&nbsp; that&nbsp; age (odds&nbsp; ratio&nbsp; 2.24,&nbsp; 95% CI:&nbsp; 1.29-3.89)&nbsp; and&nbsp; diabetes&nbsp; duration (odds ratio 1.90, 95% CI: 1.11-3.24) had significant&nbsp; association&nbsp; with&nbsp; glucose&nbsp; con- trol as shown in Table 1.&nbsp;</p>\r\n<p class="rtejustify">\r\n	DM&nbsp; knowledge&nbsp; covered&nbsp; aspects&nbsp; of diabetes&nbsp; symptom,&nbsp; dietary&nbsp; control,&nbsp; exer- cise,&nbsp; complications&nbsp; and&nbsp; risk&nbsp; factors. Mean&nbsp; DM&nbsp; knowledge&nbsp; score&nbsp; of&nbsp; the&nbsp; res- pondents&nbsp; was&nbsp; 16.96+2.25&nbsp; (maximum score&nbsp; was&nbsp; 20.0).&nbsp; Family&nbsp; support&nbsp; was assessed&nbsp; with&nbsp; regards&nbsp; to&nbsp; physical,&nbsp; emo- tional&nbsp; and&nbsp; information&nbsp; support.&nbsp; The&nbsp; ma- jority of the respondents had good family support&nbsp; in&nbsp; all&nbsp; the&nbsp; three&nbsp; aspects.&nbsp;&nbsp;&nbsp; Mean family&nbsp; support&nbsp; score&nbsp; of&nbsp; the&nbsp; respondents was&nbsp; 29.55+4.99&nbsp; (maximum&nbsp; score&nbsp; was 40.0). However, there were no significant associations&nbsp; between&nbsp; glucose&nbsp; control and&nbsp; diabetic&nbsp; knowledge&nbsp; (t=0.757,&nbsp; 95% CI:&nbsp; -3.60-0.75)&nbsp; and&nbsp; family&nbsp; support&nbsp; (t=- 0.823,&nbsp; 95%&nbsp; CI:&nbsp; -1.84-0.75)&nbsp; as&nbsp; shown&nbsp; in Table 2.</p>\r\n<p class="rtejustify">\r\n	&nbsp;Logistic&nbsp; regression&nbsp; was&nbsp; used&nbsp; in&nbsp; multi- variate&nbsp; analysis&nbsp; and&nbsp; results&nbsp; showed&nbsp; the factors&nbsp; that&nbsp; influenced&nbsp; the&nbsp; good&nbsp; glucose control&nbsp; were&nbsp; gender&nbsp; (Adjusted&nbsp; preva- lence&nbsp; odds&nbsp; ratio,&nbsp; APOR=1.75,&nbsp; 95% CI:1.02-3.00),&nbsp; age&nbsp; (APOR=2.48,&nbsp; 95% CI:1.40-4.39)&nbsp; and&nbsp; DM&nbsp; duration (APOR=2.10,&nbsp; 95%&nbsp; CI:1.21-3.64)&nbsp; as stated in Table 3.&nbsp;</p>\r\n<p class="rtejustify">\r\n	DISCUSSION</p>\r\n<p class="rtejustify">\r\n	&nbsp;Glucose control is an important aspect of diabetes management, as it is one of the risk&nbsp; factors&nbsp; for&nbsp; diabetic&nbsp; complications. Furthermore,&nbsp; it&nbsp; influences&nbsp; the&nbsp; patient&rsquo;s quality&nbsp; of&nbsp; life&nbsp; and&nbsp; increases&nbsp; the&nbsp; financial burden&nbsp; of&nbsp; diabetes&nbsp; management.&nbsp; In&nbsp; this study,&nbsp; the&nbsp; prevalence&nbsp; of&nbsp; good&nbsp; glucose control&nbsp; was&nbsp; 22.5%.&nbsp; It&nbsp; showed&nbsp; that&nbsp; the majority&nbsp; of&nbsp; the&nbsp; respondents&nbsp; had&nbsp; poor glucose&nbsp; control.&nbsp; This&nbsp; finding&nbsp; was&nbsp; similar to&nbsp; previous&nbsp; studies,&nbsp; done&nbsp; in Kulim&nbsp; and Kelantan&nbsp; in&nbsp; Malaysia,&nbsp; and&nbsp; Bangkok&nbsp; in Thailand&nbsp; which&nbsp; showed&nbsp; that&nbsp; the preva- lence&nbsp; of&nbsp; good glucose&nbsp; control&nbsp; ranged from&nbsp; 21.7%&nbsp; to 33.0%&nbsp; (Shahidan&nbsp; et al. 2003;&nbsp; Eid&nbsp; et&nbsp; al.&nbsp; 2003;&nbsp; Ngarmukos&nbsp; et al. 2006; Howteerakul et al. 2007).&nbsp;&nbsp;</p>\r\n<p class="rtejustify">\r\n	However,&nbsp; there&nbsp; were&nbsp; a few&nbsp; differences between&nbsp; this&nbsp; study&nbsp; and&nbsp; the previous ones.&nbsp; The&nbsp; previous&nbsp; studies&nbsp; used HbA1c&lt;7.0%,&nbsp; but&nbsp; this&nbsp; study&nbsp; used HbA1c&lt;6.5%&nbsp; for&nbsp; the level&nbsp; of&nbsp; good glucose&nbsp; control&nbsp; (based&nbsp; on CPG&nbsp; DM 2004).&nbsp; Another&nbsp; difference&nbsp; was&nbsp; that&nbsp; the present&nbsp; study&nbsp; involved&nbsp; only&nbsp; elderly diabetics&nbsp; who&nbsp; sought&nbsp; treatment&nbsp; at the primary&nbsp; level,&nbsp; whereas&nbsp; the previous studies&nbsp; involved&nbsp; both&nbsp; young&nbsp; and&nbsp; elderly individuals&nbsp; at the&nbsp; tertiary&nbsp; level&nbsp; (Eid&nbsp; et al. 2003; Ngarmukos et al. 2006).&nbsp;</p>\r\n<p class="rtejustify">\r\n	This&nbsp; study&nbsp; showed&nbsp; that&nbsp; males&nbsp; had&nbsp; two times&nbsp; the probability&nbsp; of&nbsp; having&nbsp; good&nbsp; glu- cose&nbsp; control&nbsp; compared&nbsp; to females.&nbsp; This finding&nbsp; was&nbsp; similar&nbsp; to&nbsp; that&nbsp; of&nbsp; Mercado&nbsp; &amp; Vargas&nbsp; (1990)&nbsp; and according&nbsp; to them, this&nbsp; was&nbsp; probably&nbsp; due&nbsp; to&nbsp; the&nbsp; fact&nbsp; that someone&nbsp; else&nbsp; prepared&nbsp; the&nbsp; food&nbsp; for&nbsp; the males,&nbsp; where&nbsp; as&nbsp; females&nbsp; prepared&nbsp; it themselves.&nbsp; Food&nbsp; preparation&nbsp; by&nbsp; some- one else has an association with glucose control&nbsp; (Epple&nbsp; et&nbsp; al.&nbsp; 2003).&nbsp; Furthermore, males&nbsp; were&nbsp; physically&nbsp; more&nbsp; active&nbsp; than females.&nbsp; Physical&nbsp; activity&nbsp; may&nbsp; reduce blood glucose level (Durak 1996).&nbsp;</p>\r\n<p class="rtejustify">\r\n	The majority&nbsp; of&nbsp; the&nbsp; respondents&nbsp; in&nbsp; this study&nbsp; were&nbsp; aged&nbsp; 60 to 69 years&nbsp; old. However when assessing the association of&nbsp; age factors&nbsp; and glucose&nbsp; control,&nbsp; it showed&nbsp; that,&nbsp; those&nbsp; who were&nbsp; 70 years old&nbsp; and&nbsp; above,&nbsp; were&nbsp; twice&nbsp; as&nbsp; likely&nbsp; to have&nbsp; good&nbsp; glucose&nbsp; control&nbsp; than&nbsp; those aged 60 to 69 years old. This finding was similar&nbsp; to several&nbsp; previous&nbsp; studies&nbsp; which found&nbsp; that&nbsp; the&nbsp; older&nbsp; age&nbsp; groups&nbsp; were more likely to have good glucose control (Trief et al. 1998; Eid et al. 2003; Choi &amp; Rankin&nbsp; 2009).&nbsp; The&nbsp; older&nbsp; patients&nbsp; are likely&nbsp; to&nbsp; practice&nbsp; a&nbsp; healthy&nbsp; diet&nbsp; and comply&nbsp; with&nbsp; their&nbsp; treatment&nbsp; compared with&nbsp; younger&nbsp; patients&nbsp; (Wahba&nbsp; &amp;&nbsp; Chang 2007).&nbsp; This&nbsp; study&nbsp; also&nbsp; found&nbsp; that&nbsp; there was&nbsp; no&nbsp; association&nbsp; between&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; glucose control&nbsp; and&nbsp; other&nbsp; socio-demographic factors, which was similar to the study by Suhaiza et al. (2004) in a primary health centre in Kelantan.&nbsp;</p>\r\n<p class="rtejustify">\r\n	Those&nbsp; who&nbsp; had&nbsp; DM&nbsp; for&nbsp; five&nbsp; years&nbsp; or less,&nbsp; were&nbsp; twice&nbsp; as&nbsp; likely&nbsp; to have&nbsp; good glucose&nbsp; control&nbsp; compared&nbsp; to&nbsp; those&nbsp; who had&nbsp; DM&nbsp; for&nbsp; more&nbsp; than five&nbsp; years,&nbsp; which was&nbsp; similar&nbsp; to study&nbsp; by&nbsp; Herraez&nbsp; et al. (1999) and Eid et al. (2003).&nbsp; The longer the&nbsp; DM&nbsp; duration,&nbsp; the&nbsp; harder&nbsp; it will&nbsp; be to control&nbsp; glucose&nbsp; level.&nbsp; This&nbsp; is&nbsp; probably due to drug resistance, resulting in higher doses&nbsp; and more&nbsp; drugs&nbsp; used in diabetes treatment&nbsp; (Benoit&nbsp; et al.&nbsp; 2005;&nbsp; Choi&nbsp; &amp; Rankin 2009).&nbsp;</p>\r\n<p class="rtejustify">\r\n	The&nbsp; commonest&nbsp; co-morbidity&nbsp; was hypertension,&nbsp; similar&nbsp; to&nbsp; a study&nbsp; by Niefield&nbsp; et al.&nbsp; (2003).&nbsp; Co-morbidities&nbsp; are challenges&nbsp; in the management&nbsp; of&nbsp; DM&nbsp; in the&nbsp; elderly&nbsp; (Crandall&nbsp; 2003).&nbsp; When assessing&nbsp; the&nbsp; association&nbsp; between glucose&nbsp; control&nbsp; and&nbsp; co-morbidities,&nbsp; this study found that there was no association between&nbsp; them.&nbsp; However,&nbsp; this&nbsp; finding contradicted&nbsp; from&nbsp; the study&nbsp; by&nbsp; Crandall (2003)&nbsp; and&nbsp; Suh&nbsp; et al.&nbsp; (2008)&nbsp; which showed&nbsp; that,&nbsp; good&nbsp; glucose&nbsp; control&nbsp; was more&nbsp; likely&nbsp; among&nbsp; those&nbsp; who&nbsp; did&nbsp; not have&nbsp; any&nbsp; co-morbidity&nbsp; compared&nbsp; to pa- tients&nbsp; with&nbsp; co-morbidities.&nbsp; The&nbsp; difference could&nbsp; be&nbsp; due&nbsp; to the small&nbsp; percentage&nbsp; of respondents&nbsp; in&nbsp; this&nbsp; study&nbsp; who&nbsp; did&nbsp; not have co-morbidities.</p>\r\n<p class="rtejustify">\r\n	Generally,&nbsp; the&nbsp; respondents&nbsp; in&nbsp; this&nbsp; study had&nbsp; a&nbsp; good&nbsp; knowledge&nbsp; score.&nbsp; The&nbsp; mean score&nbsp; was&nbsp; 16.96+2.25&nbsp; (maximum&nbsp; score was&nbsp; 20.00).&nbsp; When&nbsp; assessing&nbsp; the&nbsp; associ- ation&nbsp; of&nbsp; good&nbsp; glucose&nbsp; control&nbsp; and&nbsp; DM knowledge&nbsp; score,&nbsp; this&nbsp; study&nbsp; found&nbsp; that there was no association between glucose control&nbsp; and&nbsp; knowledge&nbsp; score,&nbsp; which&nbsp; was similar&nbsp; to&nbsp; the&nbsp; study&nbsp; by&nbsp; He&nbsp; &amp;&nbsp; Wharrad (2007). Although this present study showed that&nbsp; the&nbsp; diabetes&nbsp; knowledge&nbsp; among&nbsp; the respondents&nbsp; was&nbsp; good,&nbsp; other factors such as poor diet adherence and lack of physical activity&nbsp; are&nbsp; likely&nbsp; to&nbsp; have&nbsp; influenced&nbsp; the glucose control (Eid et al. 2003).&nbsp;&nbsp;</p>\r\n<p class="rtejustify">\r\n	For family support, the mean score was 29.55+4.99 (maximum score was 40.00).&nbsp; This&nbsp; finding&nbsp; showed&nbsp; that&nbsp; the respondents&nbsp; received&nbsp; good family support.&nbsp; However,&nbsp; when&nbsp; assessing&nbsp; the association between glucose control and family support, this study found that there was&nbsp; no association&nbsp; between&nbsp; them.&nbsp; This was in contradiction to the study by Epple et&nbsp; al.&nbsp; (2003)&nbsp; and&nbsp; Choi&nbsp; &amp; Rankin&nbsp; (2009), which found that, there&nbsp; was a significant association between family support in the nutritional&nbsp; aspect&nbsp; and&nbsp; glucose&nbsp; control. The&nbsp; difference&nbsp; could&nbsp; be&nbsp; due to&nbsp; the different&nbsp; aspects&nbsp; of&nbsp; family&nbsp; support studied in this recent study, which include physical,&nbsp; emotional&nbsp; and&nbsp; informational support.&nbsp;</p>\r\n<p class="rtejustify">\r\n	There were a few weaknesses found in this&nbsp; study.&nbsp; As&nbsp; this&nbsp; was&nbsp; a&nbsp; cross&nbsp; sectional study, the cause and effect could not be established&nbsp; by&nbsp; the&nbsp; factors&nbsp; studied.&nbsp; The respondents&nbsp; did&nbsp; not&nbsp; include&nbsp; elderly&nbsp; pa- tients&nbsp; who&nbsp; sought&nbsp; treatment&nbsp; at&nbsp; private clinics&nbsp; and&nbsp; hospitals.&nbsp; This&nbsp; will&nbsp; limit&nbsp; the generalization&nbsp; to&nbsp; all&nbsp; elderly&nbsp; diabetics. There was also the likelihood of informa- tion&nbsp; bias&nbsp; from&nbsp; the&nbsp; respondents&nbsp; as&nbsp; they might&nbsp; not&nbsp; give&nbsp; the&nbsp; true&nbsp; answer;&nbsp; either they&nbsp; have&nbsp; forgotten&nbsp; or&nbsp; refused&nbsp; to&nbsp; reveal the truth.</p>\r\n<p class="rtejustify">\r\n	CONCLUSION</p>\r\n<p class="rtejustify">\r\n	&nbsp;The&nbsp; prevalence&nbsp; of&nbsp; good&nbsp; glucose&nbsp; control in&nbsp; this&nbsp; study&nbsp; was&nbsp; still&nbsp; low.&nbsp; Elderly diabetics who were involved in this study are&nbsp; at&nbsp; high&nbsp; risk&nbsp; of&nbsp; developing&nbsp; diabetic complications. It will affect their quality of life&nbsp; and&nbsp; increases&nbsp; financial&nbsp; burden&nbsp; in diabetes management. The factors asso- ciated&nbsp; with&nbsp; good&nbsp; glucose&nbsp; control&nbsp; were gender,&nbsp; age&nbsp; and&nbsp; duration&nbsp; of&nbsp; diabetes. Therefore,&nbsp; awareness,&nbsp; exposure&nbsp; and health&nbsp; education&nbsp; on&nbsp; the&nbsp; nutritional&nbsp; as- pects,&nbsp; diabetes&nbsp; treatment&nbsp; and&nbsp; complica- tions&nbsp; should&nbsp; be&nbsp; emphasized&nbsp; at&nbsp; the&nbsp; early stage of diabetes diagnoses.&nbsp;</p>\r\n<p class="rtejustify">\r\n	ACKNOWLEDGEMENTS</p>\r\n<p class="rtejustify">\r\n	We&nbsp; would&nbsp; like&nbsp; to&nbsp; thank&nbsp; the&nbsp; Ministry&nbsp; of Health&nbsp; Malaysia,&nbsp; Kedah&nbsp; State&nbsp; Health Department,&nbsp; health&nbsp; staffs&nbsp; and respondents&nbsp; who&nbsp; were&nbsp; involved&nbsp; in&nbsp; this study.&nbsp; This&nbsp; study&nbsp; was&nbsp; supported&nbsp; by&nbsp; a research&nbsp; grant&nbsp; from&nbsp; the&nbsp; Faculty&nbsp; of Medicine,&nbsp; Universiti&nbsp; Kebangsaan Malaysia Medical Centre (FF-244-2008).</p>\r\n', 2);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(467, 464, '<p class="rtejustify">\r\n	<strong>INTRODUCTION</strong><br />\r\n	<br />\r\n	Nurses were reportedly encountering endless stress while pursuing their un- dergraduate programme (Timmins &amp; Kaliszer 2002a; Evans &amp; Kelly 2004). Their inability&nbsp; to&nbsp;&nbsp; overcome&nbsp; stressors faced at various levels during their nurs- ing education caused many to leave nursing (Glossop 2002; Last &amp; Fulbrook 2003). Research conducted over&nbsp; many years demonstrates a consistent high level of stress experienced by student nurses (Jones &amp; Johnston 1999; Gibbons et al. 2008). Nursing students&rsquo; stress was related to various academic, clinical and personal&nbsp; aspects&nbsp; encountered through- out their tertiary education (Jones &amp; Johnston&nbsp;&nbsp; 1997;&nbsp;&nbsp; Timmins&nbsp;&nbsp; &amp;&nbsp;&nbsp; Kaliszer 2002a; Lo 2002; Evans &amp; Kelly 2004; Pryjmachuk &amp; Richards 2007; Gibbons et al. 2008).</p>\r\n<p class="rtejustify">\r\n	To the best of our knowledge, there has been no study in Malaysia exploring this phenomenon among nursing students. Inthe past, few studies have examined stress&nbsp;&nbsp; &nbsp;related&nbsp;&nbsp; &nbsp;to nursing education among nursing students across all year levels (Lo 2002; Burnard et al. 2007). Therefore, the aim of this study was to examine the undergraduate nursing stu- dents&rsquo; perceptions of the intensity of per- ceived stress, the influence of demographic variables on students&rsquo; perceived stress and the relationship between students perceived stress and their aca- demic and clinical performance.</p>\r\n<p class="rtejustify">\r\n	Academic stress is&nbsp; related to&nbsp; various factors that nursing students encounter such&nbsp;&nbsp; &nbsp;as examinations/assignments (Jones &amp; Johnston 1997; Lindop 1999; Timmins &amp; Kaliszer 2002a; Deary et al. 2003; Evans &amp; Kelly 2004; Gibbons et al. 2008),&nbsp; amount of&nbsp; class-work (Evans &amp; Kelly 2004; Tully 2004) and longer study hours (Beck &amp; Srivastava 1991; Jones &amp; Johnston 1997; Lo 2002; Timmins &amp; Kaliszer 2002a). Nursing students irre- spective of the&nbsp;&nbsp; &nbsp;nursing&nbsp;&nbsp; &nbsp;programme (Bachelor and Diploma) reported similar common stressors in the studies of both Beck and Srivastava (1991) and Timmins and Kaliszer (2002a). Demographic factors which contribute to academic stress are age and gender. Academic stress is more prominent among female students with children during the period of examinations (Gibbons et al. 2008).</p>\r\n<p class="rtejustify">\r\n	Student&rsquo;s stress in the clinical setting is associated with the complexity and acuity of patient care, interacting with members of the multidisciplinary team and clinical environment. Students reported that caring for dying patients (Timmins &amp; Kaliszer 2002a; Evans &amp; Kelly 2004), critically ill&nbsp; patients (Lo&nbsp; 2002), patients diagnosed&nbsp;&nbsp; &nbsp;with HIV/AIDS&nbsp;&nbsp; &nbsp;(Jones&nbsp;&nbsp; &nbsp;&amp; Johnston&nbsp; 1997)&nbsp; and&nbsp; newborns&nbsp; (Mahat 1998) as very stressful and could lead to attrition. Student nurses also experience stress when interacting with colleagues, patients,&nbsp; other&nbsp; staff&nbsp; (Mahat&nbsp; 1998;&nbsp; Lo 2002; Timmins &amp; Kaliszer 2002a; Evans &amp;&nbsp;&nbsp; Kelly&nbsp;&nbsp; 2004)&nbsp;&nbsp; and&nbsp;&nbsp; nurse&nbsp;&nbsp; educators (Mahat 1998; Timmins &amp; Kaliszer 2002a; Magnussen &amp; Amundson 2003).</p>\r\n<p class="rtejustify">\r\n	The negative attitudes shown by the ward staff towards nursing students (Brodie et al. 2004; Evans &amp; Kelly 2004; Gibbons et al. 2008) and lack of positive relationships&nbsp; with&nbsp; nursing&nbsp; staff&nbsp; (Mahat 1998; Timmins &amp; Kaliszer 2002a) has been shown to increase student stress. In addition, the negative attitude of tutors was reported by Lindop (1989) as a con- tributing factor to&nbsp; the nursing students&rsquo; attrition (20%) from their nursing course.</p>\r\n<p class="rtejustify">\r\n	The clinical environment coupled with the use of complex equipment (Lo 2002), heavy workload, interpretation and implementation of lengthy and complex nursing care plans (Magnussen &amp; Amundson 2003) and working with patients (Mahat&nbsp; 1998;&nbsp; Jones&nbsp; &amp;&nbsp; Johnston 1997) are constant sources of student stress. Similarly, stress related to working in an understaffed environment was also reported by Brodie et al. (2004) and Lindop (1989).</p>\r\n<p class="rtejustify">\r\n	Student nurses&rsquo; stress is also related to personal factors such as&nbsp; adaptation to new&nbsp; environment, financial burden and problems related to family matters. Stu- dents have to adjust to new surroundings away&nbsp; from&nbsp; home&nbsp; (Brown&nbsp; &amp;&nbsp; Edelmann 2000; Deary et al. 2003; Pryjmachuk &amp; Richards 2007), new learning environ- ments, which highlights the difference between what is taught in the university setting and what is practiced in the clinical environment (Jones &amp; Johnston 1997; Evans &amp; Kelly 2004; Gibbons et al.2008) and adapting to new student roles (Brown &amp; Edelmann 2000; Deary et al. 2003; Pryjmachuk &amp; Richards 2007; Gibbons et al. 2008). Nursing students stress is further compounded by their financial burden as the majority of student nurses have to work to support their study (Brown&nbsp; &amp;&nbsp; Edelmann 2000).&nbsp; This results in limited free time and ultimately further&nbsp; stress&nbsp; (Pryjmachuk&nbsp; &amp;&nbsp; Richards 2007).<br />\r\n	<br />\r\n	<strong>MATERIALS AND METHODS</strong><br />\r\n	<br />\r\n	A non-experimental descriptive explora- tory survey was carried out to examine the student nurses&rsquo; level of perceived stress and factors that may influence this variable. The Student Nurse Stress Index (SNSI) developed by Jones and Johnston (1999) was used to measure student nurses stress levels. Selection of the SNSI (self-report instrument) was based on the review of the literature and review of&nbsp; the&nbsp; instrument based on the areas of study and, the reliability and validity&nbsp; characteristics&nbsp; of&nbsp; the&nbsp; instrument. The SNSI instrument used in this study was tested for reliability with the popula- tion of the present study. The Cronbach alpha&nbsp; of&nbsp; 0.87&nbsp; for&nbsp; SNSI&nbsp; and&nbsp; 0.688&nbsp; to 0.817&nbsp; for&nbsp; the&nbsp; domains&nbsp; in&nbsp; the&nbsp; present study is consistent with that reported (&alpha;&ge;0.7) by other authors (Jones &amp; Johnston 1999).The&nbsp;&nbsp; &nbsp;SNSI which consisted of 22 items clustered into four domains: academic workload, clinical concerns, personal problems and interface worries. All items used a Likert scale&nbsp; ranging&nbsp; from&nbsp; 1=not&nbsp; stressful&nbsp; to 5=extremely stressful.<br />\r\n	<br />\r\n	The research population was student nurses of Universiti Kebangsaan Malaysia (UKM). Probability sampling was used and students&rsquo; selection was based on criteria that were carefully con- sidered to represent the population under study. Bachelor of Nursing students (year 1- 4) and Diploma of Nursing students (year 1- 3) age 18 years and above were included in this study. Student nurses undergoing an&nbsp; accelerated Bachelor&nbsp; of Nursing study (2 years program) were excluded from&nbsp; this&nbsp; study.&nbsp; A&nbsp; sample of 241 nursing students returned completed questionnaires with&nbsp; a&nbsp; response rate&nbsp; of 97%.The nursing&nbsp;&nbsp; &nbsp;students of the Diploma program who participated in this study&nbsp;&nbsp; (n=172),&nbsp;&nbsp; comprise&nbsp;&nbsp; of&nbsp;&nbsp; year&nbsp;&nbsp; 1 (n=55), year 2 (n=58) and year 3 (n=59). The Bachelor of&nbsp; Nursing students who participated in this study (n=69), include year 1 (n=19), year 2 (n=18), year 3 (n=14) and year 4 (n=18).<br />\r\n	<br />\r\n	Approval to conduct the study was granted by the Research &amp; Ethics Committees of the UKM in Malaysia and Flinders University in Australia. The tool was accompanied by an information statement outlining the purpose of the survey, requesting voluntary participation and instructions on how to complete the survey and student nurse consent forms. Participants who consented&nbsp;&nbsp; to their academic and clinical results were requested to provide their student identification number.</p>\r\n<p class="rtejustify">\r\n	Data was analysed using Predictive Analytics Software (PASW) Statistics 17. Demographic data were analyzed using descriptive statistics. ANOVA and the Kruskal-Wallis test were used to deter- mine whether the&nbsp; mean&nbsp; scores&nbsp; across year levels of both the Diploma and Bachelor groups differed. Spearman Rho was used to test the relationship between perceived stress and its domains (academic load, clinical concerns, personal problems and interface worries) and demographic variables (age, gender and qualification at entry in the nursing programme) and students&rsquo; performance.<br />\r\n	<br />\r\n	<strong>RESULTS</strong><br />\r\n	<br />\r\n	In this study, both the Diploma and Bachelor groups contained a higher percentage of female than male nursing students (Table 1). The participation of Diploma nursing students is higher as compared to the Bachelor. Entry into the Bachelor programme required at least a pre-university level of education while the Diploma programme did&nbsp; not&nbsp; (Table&nbsp; 1). This difference in entry requirements is also reflected in the average age of students&nbsp; with a&nbsp; statistically significant (t=-4.798, p=0.000) larger proportion of Bachelor students being older than Di- ploma students.<br />\r\n	<br />\r\n	A significant difference in the severity of stress among the Diploma nursing students across different year levels was observed (Table 2). Post-hoc comparisons using Tukey&rsquo;s Honestly Significant Difference (HSD) test revealed that the second year students (Diploma group) claimed being more stressed than their peers in the first and third years (Table 2). However, there were no significant differences in the severity of stress perceived by the Bachelor of Nursing students across the four year levels (Table 3).<br />\r\n	<br />\r\n	Significant differences in the severity of stress related to personal problem domains of perceived stress were noted among the Diploma of Nursing students across the different years of study (Table 2). Post-hoc Tukey&rsquo;s HSD demonstrated that significantly second year students (of the&nbsp; Diploma&nbsp; programme), claimed&nbsp; that personal problems was the main domain of the perceived stress that contributed to their stress levels compared to their peers (Table 2). The main item that was perceived to cause stress in the majority of the second year Diploma students was their family members&rsquo; health&nbsp; problems (Table 2a). However, nursing students across the different years of the Bachelor programme perceived that personal problems did not cause stress to them (Table 3).</p>\r\n<p class="rtejustify">\r\n	Comparisons using the Mann-Whitney U test revealed that the student nurses in the first year of the Bachelor programme perceived that the academic load domain of the perceived stress significantly caused more stress to them than their coursemates (2nd, 3rd and 4th years) (Table 3). The main items that were reported as very stressful to the first year Bachelor students were workload of nursing education and fear of failing the nursing pro- gramme (Table 3a).<br />\r\n	<br />\r\n	Nursing students&nbsp; of&nbsp; both&nbsp; programmes, irrespective of the year of study, perceived that clinical concerns and interface worries domains of the perceived stress were&nbsp; not the major factors that contributed to their stress (Tables 2 and 3).<br />\r\n	<br />\r\n	There were no significant differences between&nbsp;&nbsp; male and female nursing students of either the Diploma and Bachelor programme in their level of stress related either to the total perceived stress or individual domains (academic load, clinical concerns, personal problems and in- terface worries) (Table 4).<br />\r\n	<br />\r\n	Older&nbsp; Bachelor&nbsp; of&nbsp; Nursing&nbsp; students (&ge;21&nbsp;&nbsp; years)&nbsp;&nbsp; perceived&nbsp;&nbsp; a&nbsp;&nbsp; significantly lower level of stress related to the academic domain only compared to their younger peers (&lt;21 years) (Table 5).&nbsp; No significant&nbsp; differences&nbsp; in&nbsp; the&nbsp; level&nbsp; of stress related to all the domains of perceived stress (academic load, clinical concerns, personal problems and interface worries) were found among the nursing students of the Diploma programme irrespective of age groups (Table 5).<br />\r\n	<br />\r\n	No significant correlations were noted between perceived stress and all its domains (clinical concerns, personal problems and interface worries) except for academic load and Cumulative Grade Point Average (CGPA) scores among the nursing students of the Diploma cohort (Table 6). A negatively significant correlation was noted between the academic load domain of the perceived stress and CGPA scores among the nursing stu- dents of&nbsp; the Diploma cohort (Table 6). Student nurses of the Diploma program who&nbsp; were able to&nbsp; overcome stress related to workload of nursing education in the classroom, difficulty of the workload of the nursing education, examinations and fear of failing nursing course ob- tained higher CGPA scores. However, no significant correlations were observed between perceived stress and its domains (academic load, clinical concerns, personal problems and interface worries) and CGPA scores among&nbsp;&nbsp; &nbsp;the Bachelor students (Table 6).<br />\r\n	<br />\r\n	Likewise, no significant correlations were&nbsp; noted&nbsp; between&nbsp; perceived&nbsp; stress and its domains (academic load, clinical concerns, personal problems and interface worries) and Grade Point Average (GPA) scores among both the Diploma and Bachelor groups (Table 6).<br />\r\n	<br />\r\n	Negative significant correlations were observed between perceived stress and all&nbsp; its&nbsp; domains (academic load, clinical concerns, personal problems and interface worries) and clinical score among the&nbsp; Diploma&nbsp; group&nbsp; (Table&nbsp; 6).&nbsp; On&nbsp; the other hand,&nbsp; positive&nbsp; significant&nbsp; correlation was observed between academic load domain (of perceived stress) alone among the Bachelor of Nursing students (Table 6).<br />\r\n	<br />\r\n	<strong>DISCUSSION</strong><br />\r\n	<br />\r\n	The nursing students of both the Diploma and Bachelor programmes in this study experienced stress related to academic components of their undergraduate nursing education. Academic load which incorporates workload, students&rsquo; difficulties in the classroom, examinations and fear of&nbsp; failing was perceived as a non-significant stressor by the Diploma students but as&nbsp;&nbsp; &nbsp;significantly stressful by first year students of the Bachelor&nbsp; programme compared&nbsp; to&nbsp; their seniors in the present study. Workload of nursing education and fear of failing the nursing programme were reported as the major stressors. Stress related to &ldquo;classwork&rdquo; of nursing education was also found to be a significant cause of stress to the 1st year students in the study by Deary et al. (2003). Stress related to workload is not&nbsp; only&nbsp; a&nbsp; frequently reported&nbsp; issue of concern among the nursing students (Evans &amp; Kelly 2004) but it is also a common issue among university students irrespective of the type of study undertaken (Pryjmachuk &amp; Richards 2007). On the contrary, Junious et al. (2010) found that the most significant stressor among senior-level nursing students (n=10) was workload of nursing education. Similarly, in a study by Jimenez et al.&nbsp;&nbsp; &nbsp;(2010),&nbsp;&nbsp; &nbsp;experienced Spanish nursing students (2nd&nbsp; &amp; 3rd&nbsp; year) reported greater stress related to aca- demic load than the novice (1st&nbsp; year) stu- dents and workload of nursing education was found to be more stressful among the second year students.<br />\r\n	<br />\r\n	The findings of Brodie et al. (2004) support the possible explanation for the higher degree of perceived stress related to workload by first year students as relating to the imbalance in the perceived and actual complexity of nursing education by the first year Bachelor nursing students as they were novices to nursing education.&nbsp; Even&nbsp; though&nbsp; the&nbsp; effect&nbsp; of stress&nbsp; related to&nbsp; workload&nbsp; was&nbsp; not&nbsp; examined in the present study, the&nbsp; study by Timmins and Kaliszer (2002b), showed that&nbsp; stress&nbsp; related&nbsp; to&nbsp; workload&nbsp;&nbsp; &nbsp;led&nbsp; to nursing students&rsquo; being absent from lectures and clinical placements.<br />\r\n	<br />\r\n	The first year Bachelor of nursing students&rsquo; fear of failing was not shared by their peers (1st year) of the Diploma cohort and their seniors of the Bachelor programme.The possible explanations for this could be due to the senior Bachelor students&rsquo; previous year/s&nbsp;&nbsp; &nbsp;achievement and awareness of&nbsp; the&nbsp; high&nbsp; chance of&nbsp; being employed upon graduation due to the current shortage and high demand for nurses in Malaysia (Edwards 2008). The senior Bachelor of Nursing students&rsquo; confidence in securing a Registered Nurse position after graduation was shared by the nursing students in the study by Norman et al. (2005). The fear of failing while studying was reported as commonly causing stress to&nbsp; nursing&nbsp; students&nbsp; (Jones&nbsp; &amp;&nbsp; Johnston 1997). The fear of failing among first year Bachelor of Nursing students could cause them to study hard to obtained good CGPA scoring&nbsp;&nbsp; &nbsp;as a negatively&nbsp;&nbsp; &nbsp;significant (p=0.000) correlation was found between age and CGPA scores among the Bachelor students in this study.<br />\r\n	&nbsp;<br />\r\n	On the other hand, perceived stress related to academic load domain was not significantly obvious among&nbsp;&nbsp; &nbsp;Diploma nursing students in this study. This finding is consistent with the findings of Burnard et al. (2008) who reported constant levels of stress related to academic aspects among the Diploma nursing students from Wales, Malta&nbsp;&nbsp; &nbsp;and Albania-Korce throughout their&nbsp;&nbsp; &nbsp;three year course. However, Tully (2004) reported significant level of workload stress among Diploma nursing&nbsp;&nbsp; &nbsp;students.&nbsp;&nbsp; &nbsp;The &ldquo;amount of classwork to learn&rdquo; and &ldquo;exams and grades&rdquo;&nbsp; of&nbsp; the&nbsp; academic&nbsp; load&nbsp; domain were&nbsp;&nbsp; reported as extremely stressful by first year Diploma students whereas &ldquo;amount of classwork to learn&rdquo; and &ldquo;fear of failing&nbsp; course&rdquo; were noted&nbsp; as&nbsp; major stressors among second year students.<br />\r\n	<br />\r\n	A&nbsp; mixture of&nbsp; negatively and&nbsp; positively significant correlations was observed between academic load domain of the SNSI instrument used in this study and clinical score among the Diploma and Bachelor nursing students respectively.<br />\r\n	Clinical concerns relating to patients&rsquo; attitudes towards student nurses and their profession, the responsibility of caring for patients, the clinical setting and students&rsquo; relationships with other health care personnel&nbsp;&nbsp; &nbsp;were explored. Nursing students in all years of both the Diploma and Bachelor of Nursing programmes did not see the clinical aspect of their nursing education as a significant stressor. These findings are consistent with those of Jones and&nbsp;&nbsp; Johnston&rsquo;s&nbsp;&nbsp; &nbsp;(1997).&nbsp;&nbsp; &nbsp;Unlike&nbsp;&nbsp; other studies (Jones &amp; Johnston 1997; Timmins &amp; Kaliszer 2002a; Lo 2002; Magnussen &amp; Amundson 2003; Evans &amp; Kelly 2004) the nurses&rsquo;&nbsp;&nbsp; &nbsp;responsibility&nbsp;&nbsp; &nbsp;for caring for patients was also perceived as non stressful in this study. In previous studies increased levels of student stress was associated with the burden of covering shifts and limited time to care for patients in understaffed wards (Brodie et al. 2004;Gibbons et al. 2008) and caring for complex patients (Jones &amp; Johnson 1997; Timmins &amp; Kaliszer 2002a; Lo 2002; Magnussen &amp; Amundson 2003; Evans &amp; Kelly 2004). Non-significant correlations were noted also between the student nurses&rsquo; clinical concerns and their clinical scores for Bachelor of Nursing students. Conversely,&nbsp;&nbsp; &nbsp;a&nbsp;&nbsp; &nbsp;negative&nbsp;&nbsp; &nbsp;significant correlation was observed between clinical concerns and the clinical score/grade for Diploma of Nursing students.<br />\r\n	<br />\r\n	The&nbsp; majority of&nbsp; the&nbsp; studies&nbsp; comparing clinical and academic related stress revealed that the academic component is the greatest source of stress to nursing students&nbsp;&nbsp; (Timmins&nbsp;&nbsp; &amp;&nbsp;&nbsp; Kaliszer&nbsp;&nbsp; 2002a; Evans &amp; Kelly 2004; Burnard et al. 2007). Contradictory results were observed in a study by Burnard et al. (2008) where Di- ploma of Nursing students from Albania- Tirana&nbsp; reported stress&nbsp; related to&nbsp; clinical aspects of their programme as greater than that related to academic aspects. In the same study however, Diploma nursing students from Brunei and Malta reported greater academic than clinical aspects related stressors. Students in two other studies also found clinical as more stressful (Lindop 1989; Beck &amp; Srivastava 1991).<br />\r\n	Students&rsquo;&nbsp; personal&nbsp;&nbsp; problems&nbsp;&nbsp; in&nbsp;&nbsp; this study were related to physical health, family and relationships with parents and other personal problems. Tully (2004) also found that personal problems were the main factor contributing to stress among second year Diploma of nursing students when compared to their&nbsp;&nbsp; &nbsp;peers.&nbsp;&nbsp; &nbsp;In particular was their concern about the physical&nbsp;&nbsp; &nbsp;health&nbsp;&nbsp; &nbsp;of family members (p=0.025) which significantly influenced their stress levels. Student&rsquo;s concern for their own physical health was not per- ceived as being stressful by the nursing students of either the Diploma or Bachelor programmes.<br />\r\n	The item &ldquo;relationships of oneself with parents&rdquo; was not perceived as stressful by either&nbsp; Diploma&nbsp; or&nbsp; Bachelor&nbsp; of&nbsp; Nursing<br />\r\n	&nbsp;<br />\r\n	students in this study. The non-significant contribution of the factor &ldquo;relationship with parents&rdquo; to stress in this study is in line with the findings of Jones and Johnston&rsquo;s (1997) among Scottish nursing students. Personal related stress is the second major source of stress after academic related&nbsp; stress&nbsp; (McLaughlin&nbsp; et&nbsp; al.&nbsp; 2008) and these two stressors were reported as the main reasons for nursing students&rsquo; absence from lectures and clinical areas (Timmins &amp; Kaliszer 2002b).<br />\r\n	Student nurse variables such as lack of free time, peer competition, schools&rsquo; responsiveness to student nurse needs and other health care personnel&rsquo;s attitudes towards student nurses were reported as non-significant concerns by students in all years of both the Diploma and Bachelor of Nursing programmes. These findings are inconsistent&nbsp;&nbsp; &nbsp;with&nbsp;&nbsp; &nbsp;those&nbsp;&nbsp; &nbsp;Jones&nbsp;&nbsp; &nbsp;and Johnston (1997), Beck and Srivastava (1991) and Tully (2004). Both Jones and Johnston (1997) and Beck and Srivastava (1991)&nbsp; reported nursing students&nbsp; stress being related to &ldquo;lack of free time&rdquo; and &ldquo;schools&rsquo; responsiveness to student needs&rdquo; and Tully (2004) found peer competition to&nbsp; be an aggravating factor contributing to distress among nursing students.<br />\r\n	<br />\r\n	Type of programme, year l of study, age, gender and qualifications on entry in the nursing programme had the potential to impact on students&rsquo; perceived stress. The findings of this study revealed no significant differences in the perceived levels of stress for nursing students in either the Diploma or Bachelor programmes. Statistically significant higher&nbsp; levels&nbsp; of&nbsp; stress&nbsp; were&nbsp; however noted among second year nursing students of the Diploma programme com- pared to their peers in the first and third year.<br />\r\n	<br />\r\n	In this study, older (&ge;21 years) students rated lower stress levels related to their academic workload domain of&nbsp; the SNSI instrument than younger (&lt;21 years) students&nbsp; of&nbsp; the&nbsp; Bachelor&nbsp; programme. This finding could not&nbsp; be&nbsp; compared with&nbsp; the findings of Gibbons et al. (2008)of increased stress for mature aged students because of the vast differences in age among the participants in the present study (19-24 years) and that of Gibbons et al. (2008) (18-42 years) . In the study by Gibbons et&nbsp; al., mature nursing students with children experienced problems coping with the academic aspects of their nursing course. The finding of this study is consistent with the finding of Misra and McKean (2000) where older undergraduate non-nursing students reported significant lower stress levels. Both male and female Diploma and Bachelor nursing students perceived no differences in stress level related&nbsp; to their academic&nbsp; work.&nbsp;&nbsp; Tully (2004), on the other hand, reported a significant perceived high stress level related to academic aspects between female and male Diploma of nursing students as there are limited studies investigating the effect of gender and academic stress comparisons are difficult. Likewise in the present study no difference in the level of academic stress was observed among Diploma nursing students holding&nbsp;&nbsp; &nbsp;different&nbsp;&nbsp; &nbsp;qualifications&nbsp;&nbsp; &nbsp; (pre- university and high school). This result is inconsistent with Tully&rsquo;s (2004) finding where students with previous qualifications scored lower academic related stress level than those with&nbsp;&nbsp; &nbsp;no previous qualifications.<br />\r\n	<br />\r\n	<strong>CONCLUSION</strong><br />\r\n	<br />\r\n	The findings of the present study provide important information for nursing education and have implications for future research. The fact that nursing education is stressful to nursing students is further supported by the findings of this study. Stress related to workload of nursing education, fear of failing the nursing programme are&nbsp; commonly reported stressors in many studies and was evident among the nursing students, in addition to stressors related to the physical health of their family members. The finding that age affects the stress level is an important finding.<br />\r\n	<br />\r\n	<strong>ACKNOWLEDGEMENTS</strong><br />\r\n	<br />\r\n	We would like to express our utmost gratitude to the Universiti Kebangsaan Malaysia Medical Centre (UKMMC) fundamental grant. We would like to thank the UKMMC, Malaysia and Flinders University, South Australia for giving us permission to conduct this study. We would also like to thank the staff and nursing students of the UKMMC for their participation and assistance.</p>\r\n', 2),
(468, 465, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Benign&nbsp; prostatic&nbsp; hyperplasia&nbsp; (BPH)&nbsp; is&nbsp; a clinical diagnosis and may be associated with&nbsp; a&nbsp; collection&nbsp; of&nbsp; urinary&nbsp; symptoms, known&nbsp; as&nbsp; lower&nbsp; urinary&nbsp; tract&nbsp; symptoms (LUTS).&nbsp; LUTS&nbsp; may&nbsp; be&nbsp; divided&nbsp; into&nbsp; void-ing&nbsp; (obstructive)&nbsp; and&nbsp; storage&nbsp; (irritative) symptoms.&nbsp; These&nbsp; symptoms&nbsp; experi- enced&nbsp; by&nbsp; patients&nbsp; can&nbsp; be&nbsp; assessed through&nbsp; the&nbsp; International&nbsp; Prostate Symptom&nbsp; Score&nbsp; (IPSS).&nbsp; The&nbsp; IPSS questionnaire&nbsp; has&nbsp; been&nbsp; validated&nbsp; in Malaysia&nbsp; (Quek&nbsp; et&nbsp; al.&nbsp; 2001). Nevertheless,&nbsp; LUTS&nbsp; are&nbsp; not&nbsp; specific&nbsp; for BPH alone&nbsp; as there&nbsp; are&nbsp; other conditions which may also be associated with LUTS such&nbsp; as&nbsp; urinary&nbsp; tract&nbsp; infections,&nbsp; prostate carcinoma,&nbsp; ureteric&nbsp; stones&nbsp; and&nbsp; chronic prostatitis (Cheah et al. 2003).&nbsp;</p>\r\n<p>\r\n	The Ministry of Health of Malaysia cited that BPH affects about 50% of men aged 60&nbsp; years&nbsp; and&nbsp; above&nbsp; (Ministry&nbsp; of&nbsp; Health Malaysia 2002). In Kuala Lumpur, 18.9% and 39.6%&nbsp; of the men were found to be severely&nbsp; and&nbsp; moderately&nbsp; symptomatic, respectively. The prevalence of moderate to&nbsp; severe&nbsp; LUTS&nbsp; in&nbsp; Malays,&nbsp; Chinese&nbsp; and Indians&nbsp; were&nbsp; 70%,&nbsp; 59%&nbsp; and 50%&nbsp; re- spectively (Teh 2001). In Penang, 80% of men reported some degree of LUTS, but the&nbsp; prevalence&nbsp; of&nbsp; moderate&nbsp; to severe LUTS was only 6.2% (Mariappan &amp; Wooi 2006).&nbsp;</p>\r\n<p>\r\n	From&nbsp; this&nbsp; study,&nbsp; we&nbsp; aim&nbsp; to&nbsp; determine the&nbsp; prevalence&nbsp; of&nbsp; LUTS&nbsp; among&nbsp; male outpatients&nbsp; in&nbsp; Universiti&nbsp; Kebangsaan Malaysia&nbsp; Medical&nbsp; Center&nbsp; (UKMMC),&nbsp; be- sides&nbsp; assessing&nbsp; their&nbsp; level&nbsp; of&nbsp; awareness towards BPH.&nbsp;</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS&nbsp; </strong></p>\r\n<p>\r\n	This&nbsp; cross-sectional&nbsp; study&nbsp; was&nbsp; con- ducted in the surgical and medical clinics of UKMMC, from 20th October, 2009 until 20th&nbsp; May,&nbsp; 2010.&nbsp; Male&nbsp; outpatients&nbsp; were selected&nbsp; through&nbsp; convenient&nbsp; sampling. Inclusion criteria include patients of age &ge; 40&nbsp; years&nbsp; in&nbsp; stable&nbsp; clinical&nbsp; condition&nbsp; with subject&nbsp; literacy&nbsp; (able&nbsp; to&nbsp; understand&nbsp; and respond to questions).&nbsp;</p>\r\n<p>\r\n	The&nbsp; questionnaire&nbsp; used&nbsp; in&nbsp; this&nbsp; study consists&nbsp; of&nbsp; demographic&nbsp; data,&nbsp; questions related&nbsp; to&nbsp; knowledge,&nbsp; attitude&nbsp; and prac- tice&nbsp; on BPH&nbsp; and&nbsp; the International&nbsp; Pros- tate Symptom Score (IPSS). The aware- ness&nbsp; (knowledge,&nbsp; attitude&nbsp; and practice) of&nbsp; the outpatients&nbsp; were&nbsp; assessed&nbsp; in this study&nbsp; via&nbsp; questionnaire&nbsp; (Table&nbsp; 1).&nbsp; They were classified into low (n=146) and high (n=74)&nbsp; education&nbsp; levels,&nbsp; with&nbsp; low&nbsp; educa- tion&nbsp; defined&nbsp; as&nbsp; SPM&nbsp; qualification&nbsp; and below&nbsp; while&nbsp; high&nbsp; education&nbsp; was&nbsp; defined as STPM qualification and above.</p>\r\n<p>\r\n	A&nbsp; Medtronic&copy;&nbsp; Uroflow&nbsp; Recorder&nbsp; was used&nbsp; to&nbsp; evaluate&nbsp; the&nbsp; function&nbsp; of&nbsp; the&nbsp; uri- nary&nbsp; tract&nbsp; and&nbsp; determine&nbsp; the&nbsp; severity&nbsp; of BPH.&nbsp; BladderScan&reg;&nbsp; BVI&nbsp; 3000&nbsp; was&nbsp; used to determine the post-void urine volume in the&nbsp; bladder.&nbsp; Mid-stream&nbsp; urine&nbsp; samples were collected for analysis on haematuria, proteinuria&nbsp; and&nbsp; nitrite&nbsp; for&nbsp; urinary&nbsp; tract infections using Combur<sup>10</sup> Test&reg; dipsticks.</p>\r\n<p>\r\n	For the uroflowmetry results to be valid, the voided urine volume must be 150 mL or greater (Ezz el Din et al. 1996). Based on&nbsp; this&nbsp; criterion,&nbsp; 118&nbsp; respondents&nbsp; were excluded.</p>\r\n<p>\r\n	The&nbsp; statistical&nbsp; analyses&nbsp; of&nbsp; the&nbsp; study were&nbsp; done using&nbsp; Statistical Package for the&nbsp; Social&nbsp; Sciences&nbsp; (SPSS)&nbsp; version 17.0.&nbsp; The&nbsp; association&nbsp; between&nbsp; IPSS score&nbsp; and&nbsp; age&nbsp; group&nbsp; as&nbsp; well&nbsp; as&nbsp; age group&nbsp; and&nbsp; uroflowmetry&nbsp; were&nbsp; analyzed by&nbsp; repeated&nbsp; measures&nbsp; analysis&nbsp; of variance&nbsp; (ANOVA).&nbsp; The&nbsp; level&nbsp; of significance&nbsp; was&nbsp; set&nbsp; at&nbsp; 0.05.&nbsp; This&nbsp; study was&nbsp; approved&nbsp; by&nbsp; the&nbsp; Ethics&nbsp; Committee of&nbsp; Universiti&nbsp; Kebangsaan&nbsp; Malaysia Medical Centre (FF-303-2009).&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS&nbsp; </strong></p>\r\n<p>\r\n	<em>Demographic Profile&nbsp; </em></p>\r\n<p>\r\n	A total of 220 male outpatients were sur- veyed in the Medical and Surgical Clinics of&nbsp; UKMMC,&nbsp; all&nbsp; of&nbsp; whom&nbsp; were&nbsp; 40&nbsp; years old and above with a mean age of 61&plusmn;10 years.&nbsp; A&nbsp; total&nbsp; of&nbsp; 51.8%&nbsp; of&nbsp; the&nbsp; respon- dents&nbsp; were&nbsp; Chinese;&nbsp; followed&nbsp; by&nbsp; 36.8% Malays,&nbsp; 9.1%&nbsp; Indians&nbsp; and&nbsp; 2.3%&nbsp; other races.&nbsp; The&nbsp; baseline&nbsp; characteristics&nbsp; are given in Table 2.&nbsp;</p>\r\n<p>\r\n	<em>Prevalence&nbsp; of&nbsp; Lower&nbsp; Urinary&nbsp; Tract Symptoms (LUTS)&nbsp; </em></p>\r\n<p>\r\n	Among&nbsp; men&nbsp; aged&nbsp; 40&nbsp; and&nbsp; above,&nbsp; the prevalence of significant LUTS (including moderately&nbsp; symptomatic&nbsp; and&nbsp; severely symptomatic)&nbsp; is&nbsp; 42.7%.&nbsp; According&nbsp; to ethnicity, 45.4% of Malays, 43.0% of Chi- nese and 40.0% of Indians were found to have significant LUTS (Table 3).&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The&nbsp; severity&nbsp; of&nbsp; LUTS&nbsp; was&nbsp; analyzed&nbsp; in relation&nbsp; to&nbsp; their&nbsp; age&nbsp; group.&nbsp; The&nbsp; mean IPSS&nbsp; score&nbsp; increased&nbsp; with&nbsp; age,&nbsp; from 5.32&plusmn;5.78&nbsp; in&nbsp; the&nbsp; 40&nbsp; to&nbsp; 49&nbsp; years&nbsp; age group to 10.89&plusmn;8.98 in the more than 70 years&nbsp; age&nbsp; group.&nbsp; This&nbsp; increase&nbsp; in&nbsp; mean IPSS&nbsp; score&nbsp; with&nbsp; age&nbsp; was&nbsp; found&nbsp; to&nbsp; be significant&nbsp; by&nbsp; ANOVA,&nbsp; in&nbsp; which&nbsp; p=0.005 at 95% confidence level (Table 4).</p>\r\n<p>\r\n	The prevalence of individual symptoms of&nbsp; IPSS&nbsp; (scores&nbsp; 1&ndash;5)&nbsp; are&nbsp; determined from the study sample, whereby the three most commonly found LUTS are nocturia (78.2%),&nbsp; re-voiding&nbsp; within&nbsp; two&nbsp; hours (58.2%)&nbsp; and&nbsp; incomplete&nbsp; emptying&nbsp; of bladder (44.6%) (Figure 1).</p>\r\n<p>\r\n	The&nbsp; quality&nbsp; of&nbsp; life&nbsp; reported&nbsp; by&nbsp; the re- spondents&nbsp; is&nbsp; classified&nbsp; according&nbsp; to&nbsp; the severity&nbsp; of&nbsp; symptoms&nbsp; based&nbsp; on IPSS. The level of satisfaction with their urinary condition&nbsp; is&nbsp; generally&nbsp; higher&nbsp; among&nbsp; the mildly&nbsp; symptomatic&nbsp; compared&nbsp; to&nbsp; the&nbsp; se- verely&nbsp; symptomatic&nbsp; respondents&nbsp; (Table 5).&nbsp; Uroflowmetry Results&nbsp; The mean voided urine volume for all the subjects (n=102) are 278&plusmn;122 mL with a mean&nbsp; Qmax&nbsp; of&nbsp; 19.8&plusmn;11.5&nbsp; mL/s&nbsp; and mean Qave of 10.8&plusmn;5.2 mL/s. The mean residual&nbsp; urine&nbsp; volume&nbsp; as&nbsp; determined&nbsp; by bladder scan was 41&plusmn;14 mL.&nbsp;</p>\r\n<p>\r\n	The&nbsp; mean&nbsp; values&nbsp; for&nbsp; both&nbsp; Qmax&nbsp; and Qave&nbsp; decreased&nbsp; with&nbsp; every&nbsp; 10 years&nbsp; in- crease&nbsp; in&nbsp; age,&nbsp; and&nbsp; these&nbsp; were&nbsp; found&nbsp; to be&nbsp; statistically&nbsp; significant&nbsp; associations (p=0.039 and 0.001 respectively). On the other&nbsp; hand,&nbsp; the&nbsp; postvoid&nbsp; residual&nbsp; urine volume&nbsp; rises&nbsp; significantly&nbsp; with&nbsp; age, with p=0.037 (Table 6).</p>\r\n<p>\r\n	<em>Urinalysis Results&nbsp; </em></p>\r\n<p>\r\n	The&nbsp; prevalence&nbsp; of&nbsp; urine&nbsp; abnormalities such&nbsp; as&nbsp; glycosuria,&nbsp; proteinuria&nbsp; and haematuria&nbsp; were&nbsp; determined&nbsp; by&nbsp; urine dipstick&nbsp; analysis.&nbsp; The&nbsp; most&nbsp; common&nbsp; uri- nary&nbsp; findings&nbsp; among&nbsp; male&nbsp; outpatients&nbsp; of UKMMC&nbsp; were&nbsp; glycosuria&nbsp; (23.6%),&nbsp; fol- lowed&nbsp; by&nbsp; proteinuria&nbsp; (11.4%).&nbsp; The prevalence&nbsp; of&nbsp; haematuria&nbsp; was&nbsp; found&nbsp; to be only 1.8% (Figure 2).&nbsp; Knowledge,&nbsp; Attitude&nbsp; &amp;&nbsp; Practice&nbsp; (KAP)&nbsp; In Relation To BPH&nbsp; More&nbsp; than&nbsp; half&nbsp; (59.5%)&nbsp; of&nbsp; the&nbsp; respondents have&nbsp; not&nbsp; heard&nbsp; of&nbsp; benign&nbsp; prostatic&nbsp; hyper- plasia before. Most of the respondents were able to answer only five questions correctly (34.1%).&nbsp;</p>\r\n<p>\r\n	From&nbsp; Figure&nbsp; 3, the&nbsp; majority&nbsp; of&nbsp; the re- spondents&nbsp; were&nbsp; able&nbsp; to&nbsp; answer&nbsp; five questions&nbsp; correctly;&nbsp; however,&nbsp; the&nbsp; per- centage of respondents with high educa- tion level (39.19%) was much higher than those&nbsp; with&nbsp; lower&nbsp; education&nbsp; level (31.51%).&nbsp; Conversely,&nbsp; the percentage&nbsp; of respondents who were unable to answer at&nbsp; least&nbsp; three&nbsp; questions&nbsp; correctly&nbsp; were found&nbsp; to be greater&nbsp; in the lower education&nbsp; group&nbsp; compared&nbsp; to the&nbsp; higher education group.</p>\r\n<p>\r\n	Over&nbsp; 78.2%&nbsp; of&nbsp; the&nbsp; respondents&nbsp; would seek medical attention if they had LUTS. The&nbsp; reasons&nbsp; commonly&nbsp; given&nbsp; by&nbsp; those who do not want to seek treatment were that LUTS was a part of the normal age- ing process and that LUTS did not affect their quality of life. In addition, there were 15.9%&nbsp; of&nbsp; respondents&nbsp; who&nbsp; would&nbsp; also seek traditional treatment for LUTS.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION&nbsp; </strong></p>\r\n<p>\r\n	Our&nbsp; study&nbsp; population&nbsp; consists&nbsp; of&nbsp; three major&nbsp; ethnic&nbsp; groups&nbsp; in&nbsp; our&nbsp; country,&nbsp; i.e. Malays,&nbsp; Chinese&nbsp; and&nbsp; Indians,&nbsp; as&nbsp; well&nbsp; as other minority races. The Chinese (51.8%) made&nbsp; up&nbsp; the&nbsp; highest&nbsp; proportion&nbsp; of&nbsp; our study&nbsp; population,&nbsp; followed&nbsp; by&nbsp; Malays (36.8%) and Indians (9.1%). This could be due&nbsp; to&nbsp; the&nbsp; relatively&nbsp; higher&nbsp; proportion&nbsp; of Chinese population in the study area. The prevalence&nbsp; of&nbsp; symptomatic&nbsp; LUTS&nbsp; based on&nbsp; a&nbsp; symptom&nbsp; ind&ge; ex&nbsp; 8&nbsp; (including moderately&nbsp; symptomatic&nbsp; and&nbsp; severely symptomatic)&nbsp; in&nbsp; our&nbsp; study&nbsp; was&nbsp; 42.7% among&nbsp; men&nbsp; of&nbsp; all&nbsp; races.&nbsp; In&nbsp; comparison with the study conducted in Hospital Kuala Lumpur&nbsp; (Ministry&nbsp; of&nbsp; Health&nbsp; Malaysia 2002),&nbsp; our&nbsp; results&nbsp; showed&nbsp; a&nbsp; lower prevalence&nbsp; among&nbsp; all&nbsp; the&nbsp; races,&nbsp; with Malays&nbsp; at&nbsp; 45.4%,&nbsp; Chinese&nbsp; 43.0%,&nbsp; and Indians 40.0%. This could be explained by the&nbsp; younger&nbsp; age&nbsp; range&nbsp; in&nbsp; our&nbsp; study population,&nbsp; with&nbsp; a&nbsp; minimum&nbsp; age&nbsp; which was ten years lower than their study.&nbsp;</p>\r\n<p>\r\n	The most&nbsp; important&nbsp; risk factor&nbsp; for&nbsp; BPH is age, being significantly associated with a&nbsp; higher&nbsp; IPSS&nbsp; score&nbsp; (more&nbsp; severe LUTS),&nbsp; lower&nbsp; maximum&nbsp; urine&nbsp; flow&nbsp; rate (Qmax) and higher post-void residual urine volume.&nbsp; Similar&nbsp; findings&nbsp; have&nbsp; been&nbsp; reported&nbsp; by&nbsp; Homma&nbsp; et&nbsp; al.&nbsp; (1997).&nbsp; This study involving Asian men showed a pro- gressing&nbsp; trend&nbsp; of&nbsp; 18%,&nbsp; 29%,&nbsp; 40%,&nbsp; and 56%&nbsp; BPH&nbsp; prevalence&nbsp; in&nbsp; the&nbsp; age&nbsp; groups of 40 to 49, 50 to 59, 60 to 69, and 70 to 79&nbsp; years,&nbsp; respectively.&nbsp; Our&nbsp; results showed&nbsp; a&nbsp; significant&nbsp; increasing&nbsp; trend&nbsp; for the mean IPSS score with every 10 year increase&nbsp; in&nbsp; age&nbsp; (p=0.005),&nbsp; which&nbsp; is&nbsp; also consistent&nbsp; with&nbsp; another&nbsp; study&nbsp; in&nbsp; Taiwan (sample&nbsp; size=100;&nbsp; p=0.02)&nbsp; (Liu&nbsp; et&nbsp; al. 2004).&nbsp; However,&nbsp; a&nbsp; study&nbsp; in&nbsp; Singapore showed&nbsp; contrasting&nbsp; results&nbsp; with&nbsp; a decreasing&nbsp; trend&nbsp; of&nbsp; median&nbsp; IPSS&nbsp; score with&nbsp; age.&nbsp; This&nbsp; is&nbsp; probably&nbsp; due&nbsp; to&nbsp; the difficulty&nbsp; in&nbsp; quantifying&nbsp; symptoms&nbsp; and cultural&nbsp; differences&nbsp; among&nbsp; the&nbsp; older patients in Singapore (Loh et al. 2009). Our&nbsp; respondents&rsquo;&nbsp; urodynamics&nbsp; were&nbsp; also assessed&nbsp; by&nbsp; uroflowmetry.&nbsp; The&nbsp; mean maximum&nbsp; urine&nbsp; flow&nbsp; rate&nbsp; (Qmax)&nbsp; was 19.8&plusmn;11.5 mL/s. Both Qmax and Qave have been shown to progressively decline with advancing&nbsp; age,&nbsp; which&nbsp; was&nbsp; consistent&nbsp; with the increase in severity of LUTS with age.&nbsp; The&nbsp; residual&nbsp; urine&nbsp; volume&nbsp; showed an&nbsp; increasing&nbsp; trend&nbsp; in&nbsp; relation&nbsp; to&nbsp; age&nbsp; as this&nbsp; indicated&nbsp; an&nbsp; increase&nbsp; in&nbsp; urinary&nbsp; re- tention&nbsp; as&nbsp; a&nbsp; result&nbsp; of&nbsp; bladder&nbsp; outlet&nbsp; obstruction.</p>\r\n<p>\r\n	Aside&nbsp; from&nbsp; BPH,&nbsp; there&nbsp; are&nbsp; many urological&nbsp; as&nbsp; well&nbsp; as&nbsp; non-urological&nbsp; dis- eases&nbsp; that&nbsp; present&nbsp; with&nbsp; LUTS&nbsp; including prostate&nbsp; cancer,&nbsp; prostatitis,&nbsp; bladder&nbsp; can- cer,&nbsp; bladder&nbsp; stones,&nbsp; overactive&nbsp; bladder, urinary&nbsp; tract&nbsp; infections,&nbsp; cystitis,&nbsp; primary bladder&nbsp; neck&nbsp; hypertrophy,&nbsp; urethritis&nbsp; and diabetes&nbsp; mellitus.&nbsp; From&nbsp; our&nbsp; study,&nbsp; the two&nbsp; most&nbsp; commonly&nbsp; reported&nbsp; LUTS&nbsp; are nocturia&nbsp; and&nbsp; frequency.&nbsp; However,&nbsp; they are non-specific and can also be caused by&nbsp; other&nbsp; diseases&nbsp; as&nbsp; well,&nbsp; for&nbsp; example urinary tract infections and diabetes mel- litus.&nbsp; The&nbsp; presence&nbsp; of&nbsp; glycosuria&nbsp; in&nbsp; our study&nbsp; (23.6%)&nbsp; is&nbsp; suggestive&nbsp; of&nbsp; diabetes mellitus&nbsp; while&nbsp; haematuria&nbsp; (1.8%)&nbsp; and nitrites&nbsp; (6.8%)&nbsp; are&nbsp; indicative&nbsp; of&nbsp; urinary tract infection. Although these symptoms are&nbsp; non-specific,&nbsp; the detection&nbsp; and&nbsp; man- agement&nbsp; of&nbsp; these&nbsp; secondary&nbsp; causes&nbsp; of LUTS&nbsp; provide&nbsp; us&nbsp; with&nbsp; another&nbsp; approach towards&nbsp; reducing&nbsp; the&nbsp; high&nbsp; prevalence&nbsp; of LUTS among the population.&nbsp;</p>\r\n<p>\r\n	We also found that those whose quality of&nbsp; life&nbsp; was&nbsp; adversely&nbsp; affected&nbsp; by&nbsp; LUTS (67.5%),&nbsp; mostly&nbsp; presented&nbsp; with&nbsp; significant&nbsp; urinary&nbsp; symptoms,&nbsp; as&nbsp; compared to those (87.4%) who presented with mild symptoms or were asymptomatic. This is consistent&nbsp; with&nbsp; a study&nbsp; done by&nbsp; Teh (2001),&nbsp; which found that there&nbsp; is a good correlation between the severity of LUTS with&nbsp; the&nbsp; quality&nbsp; of&nbsp; life&nbsp; (r=0.69,&nbsp; p&lt;0.001). Calais&nbsp; Da&nbsp; Silva&nbsp; et&nbsp; al.&nbsp; (1997)&nbsp; found&nbsp; that the&nbsp; areas&nbsp; of&nbsp; life&nbsp; most&nbsp; affected&nbsp; by&nbsp; BPH were daily activities due to frequency and sleep interrupted by nocturia.&nbsp;</p>\r\n<p>\r\n	A&nbsp; total&nbsp; of&nbsp; 34.1%&nbsp; of&nbsp; respondents&nbsp; were able&nbsp; to&nbsp; answer&nbsp; five&nbsp; out&nbsp; of&nbsp; six&nbsp; questions correctly. Since our study was conducted in&nbsp; the&nbsp; surgical&nbsp; and&nbsp; medical&nbsp; clinics&nbsp; of UKMMC,&nbsp; our&nbsp; respondents&nbsp; have&nbsp; either been&nbsp; made&nbsp; aware&nbsp; of&nbsp; BPH&nbsp; by&nbsp; their&nbsp; doc- tors&nbsp; or&nbsp; have&nbsp; read&nbsp; about&nbsp; it&nbsp; from&nbsp; the&nbsp; bro- chures or posters in the clinic. Hence, we recommend&nbsp; that&nbsp; a&nbsp; study&nbsp; should&nbsp; be&nbsp; car- ried out in the general population so that the data generated can better reflect the level of awareness in our community.&nbsp;</p>\r\n<p>\r\n	Comparing the level of knowledge about BPH between the higher education group and&nbsp; lower&nbsp; education&nbsp; group,&nbsp; as&nbsp; expected, the&nbsp; higher&nbsp; education&nbsp; group&nbsp; had&nbsp; a&nbsp; higher level&nbsp; of&nbsp; knowledge&nbsp; of&nbsp; the&nbsp; disease, whereas the lower education group had a lower&nbsp; level&nbsp; of&nbsp; knowledge.&nbsp; However, among those&nbsp; who answered all six ques- tions correctly, the results&nbsp; showed&nbsp; a con- trasting higher proportion of lower educa- tion group. This might be explained by the fact&nbsp; that&nbsp; these&nbsp; subjects&nbsp; are&nbsp; afflicted&nbsp; with BPH&nbsp; and&nbsp; hence,&nbsp; understood&nbsp; the&nbsp; disease after being educated by their doctors.&nbsp;</p>\r\n<p>\r\n	The&nbsp; last&nbsp; part&nbsp; of&nbsp; the&nbsp; assessment&nbsp; was about&nbsp; the&nbsp; health-seeking&nbsp; behaviour among&nbsp; those&nbsp; who&nbsp; had&nbsp; LUTS.&nbsp; We&nbsp; found that&nbsp; most&nbsp; (78.2%)&nbsp; patients&nbsp; with&nbsp; LUTS would&nbsp; seek&nbsp; medical&nbsp; attention&nbsp; and&nbsp; treat- ment.&nbsp; In&nbsp; addition,&nbsp; traditional&nbsp; medication was&nbsp; still&nbsp; regarded&nbsp; as&nbsp; an&nbsp; alternative treatment&nbsp; for&nbsp; LUTS&nbsp; by&nbsp; a&nbsp; small&nbsp; group&nbsp; of people&nbsp; (15.9%),&nbsp; especially&nbsp; among&nbsp; the elderly, as they still place enormous faith in traditional medicine as part and parcel of&nbsp; their&nbsp; culture&nbsp; and&nbsp; belief.&nbsp; In&nbsp; order&nbsp; to promote&nbsp; appropriate&nbsp; health-seeking&nbsp; be- haviour, awareness campaigns should be carried out in order to better educate the public&nbsp; on&nbsp; BPH,&nbsp; including&nbsp; its&nbsp; prevalence, signs&nbsp; and&nbsp; symptoms,&nbsp; treatment&nbsp; options, as&nbsp; well&nbsp; as&nbsp; its&nbsp; complications&nbsp; if&nbsp; left&nbsp; untreated.&nbsp;</p>\r\n<p>\r\n	<strong>CONCLUSION </strong></p>\r\n<p>\r\n	&nbsp;There&nbsp; is&nbsp; a&nbsp; high&nbsp; prevalence&nbsp; of&nbsp; LUTS among male outpatient attendees in Uni- versiti&nbsp; Kebangsaan&nbsp; Malaysia&nbsp; Medical Centre.&nbsp; The&nbsp; level&nbsp; of&nbsp; awareness&nbsp; is&nbsp; satis- factory. However, a more concerted effort is&nbsp; needed&nbsp; to&nbsp; screen&nbsp; patients&nbsp; in&nbsp; the&nbsp; hos- pital for this problem and provide the op- timal care for them, targeting mainly men above&nbsp; the&nbsp; age&nbsp; of&nbsp; forty.&nbsp; However,&nbsp; lower urinary&nbsp; tract&nbsp; symptoms&nbsp; are&nbsp; not&nbsp; synony- mous&nbsp; with&nbsp; BPH&nbsp; and&nbsp; men&nbsp; only.&nbsp; Further studies are also needed to look into lower urinary tract symptoms among women.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS </strong></p>\r\n<p>\r\n	&nbsp;We&nbsp; would&nbsp; like&nbsp; to&nbsp; thank&nbsp; the&nbsp; Faculty&nbsp; of Medicine,&nbsp; UKMMC&nbsp; for&nbsp; allowing&nbsp; us&nbsp; to conduct&nbsp; this&nbsp; research.&nbsp; In&nbsp; addition,&nbsp; the cooperation&nbsp; and&nbsp; help&nbsp; given&nbsp; by&nbsp; Dr.&nbsp; S. Selvalingam,&nbsp; nurses&nbsp; of&nbsp; UKMMC, volunteers&nbsp; and&nbsp; friends&nbsp; are&nbsp; highly appreciated.&nbsp; We&nbsp; would&nbsp; also&nbsp; like&nbsp; to extend&nbsp; our&nbsp; gratitude&nbsp; to&nbsp; those&nbsp; who&nbsp; are directly&nbsp; or&nbsp; indirectly&nbsp; involved&nbsp; in&nbsp; this research.&nbsp;<strong> </strong></p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(470, 467, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Stroke has become one of the major public health&nbsp; problems&nbsp; worldwide&nbsp; with&nbsp; 795,000 cases&nbsp; of&nbsp; newly&nbsp; diagnosed&nbsp; stroke&nbsp; reported yearly.&nbsp; Two&nbsp; thirds&nbsp; of&nbsp; the&nbsp; stroke&nbsp; survivors will&nbsp; have&nbsp; neurological&nbsp; and&nbsp; functional&nbsp; im- pairments&nbsp; that&nbsp; render&nbsp; them&nbsp; dependent&nbsp; in performing functional tasks and activities of daily living (Sudlow &amp; Warlow 2009).&nbsp;</p>\r\n<p>\r\n	The last two decades has witnessed the emergence&nbsp; of new medical&nbsp; advances namely&nbsp; better acute medical&nbsp; care and the availability&nbsp; of reversal agents for stroke (Dobkin&nbsp; 2004; Johansson 2010). These therapies&nbsp; were however either minimal&nbsp; or not&nbsp; easily accessible to all patients. Re- habilitation intervention remains a practical option for stroke care, providing therapeutic interventions&nbsp; that may minimize disability, thus&nbsp; helping in achieving independence and reducing social costs.</p>\r\n<p>\r\n	Recent&nbsp; evidence suggests the benefit of structured&nbsp; rehabilitation in improving&nbsp; post- stroke outcomes in stroke survivors (Stroke Unit Trialists&rsquo; Collaboration 2007; Aziz et al. 2008;&nbsp; Outpatient Service Trialists&rsquo; 2003). There is also consensus for the need of re- assessments&nbsp; and further targeted rehabili- tation for stroke survivors who report resid- ual&nbsp; impairment&nbsp; after completion&nbsp; of initial rehabilitation&nbsp; (Canadian Stroke Strategy 2006;&nbsp; Intercollegiate Stroke Working&nbsp; Party 2008).&nbsp; However, the practicality&nbsp; of imple- menting a seamless rehabilitation interven- tion&nbsp; from&nbsp; in-patient to the community&nbsp; re- mains elusive. Rehabilitation of post-stroke patients&nbsp; remains&nbsp; fragmented&nbsp; in many places.&nbsp; Many stroke patients after being discharged&nbsp; from&nbsp; in-patient care receive either single rehabilitation care intervention such&nbsp; as physiotherapy&nbsp; or occupational therapy,&nbsp; or transferred directly to commu- nity with minimal rehabilitation intervention. Another&nbsp; setback with post-stroke rehabili- tation is lack of regular assessments during rehabilitation&nbsp; intervention to assess pa- tients&rsquo;&nbsp; progress or the lack of it. The main challenge remains on how to set up a ser- vice&nbsp; that not only provides a structured rehabilitation&nbsp; intervention in an out-patient set-up, but the intervention that needs to be in&nbsp; place to accommodate&nbsp; the variable de- gree of impairments, function and needs of the stroke patients.</p>\r\n<p>\r\n	Realizing&nbsp; these deficiencies,&nbsp; we initiated a&nbsp; service of combined-stroke&nbsp; rehabilitation clinic&nbsp; (CSRC) with a multi-disciplinary&nbsp; ap- proach&nbsp; to stroke patients attending outpa- tient&nbsp; rehabilitation. With&nbsp; the aims&nbsp; of mini- mizing&nbsp; impairments&nbsp; and maximizing&nbsp; func- tion,&nbsp; all stroke patients were registered to this clinic as soon as they were discharged from the wards. The CSRC team consisted of a stroke rehabilitation specialist, physio- therapist,&nbsp; occupational&nbsp; therapist,&nbsp; speech and language therapist and medical social worker&nbsp; who&nbsp; met&nbsp; on&nbsp; a&nbsp; weekly&nbsp; basis.&nbsp; The main feature of the CSRC was the shared discussion&nbsp; among&nbsp; team&nbsp; members&nbsp; and patients&nbsp; in&nbsp; deciding&nbsp; on&nbsp; further&nbsp; goals&nbsp; and intervention.&nbsp; Post-stroke&nbsp; patients&nbsp; were designed&nbsp; a&nbsp; set&nbsp; of&nbsp; individualized&nbsp; programs which&nbsp; was&nbsp; problem-based&nbsp; approach through&nbsp; task-specific&nbsp; activities,&nbsp; and&nbsp; were monitored&nbsp; regularly&nbsp; until&nbsp; either&nbsp; completion of&nbsp; a&nbsp; two-year&nbsp; program,&nbsp; or&nbsp; able&nbsp; to&nbsp; be&nbsp; discharged to the community, whichever was earliest.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS </strong></p>\r\n<p>\r\n	<em>Combined Stroke Rehabilitation Clinic&nbsp; </em></p>\r\n<p>\r\n	The CSRC was started in 2008 as an ad- junct&nbsp; service&nbsp; to&nbsp; the&nbsp; existing&nbsp; rehabilitation services in the Universiti Kebangsaan Ma- laysia&nbsp; Medical&nbsp; Centre&nbsp; (UKMMC),&nbsp; which&nbsp; is a tertiary teaching hospital in Malaysia that covers&nbsp; approximately&nbsp; 750,000&nbsp; urban&nbsp; and semi-urban&nbsp; populations&nbsp; within&nbsp; the&nbsp; Klang Valley.&nbsp; The&nbsp; weekly&nbsp; clinic&nbsp; reviewed&nbsp; all stroke&nbsp; patients&nbsp; receiving&nbsp; rehabilitation&nbsp; in- terventions in the unit. All patients were first seen&nbsp; when&nbsp; they&nbsp; had&nbsp; received&nbsp; at&nbsp; least&nbsp; 6 sessions&nbsp; of&nbsp; the&nbsp; first&nbsp; cycle&nbsp; of&nbsp; rehabilitation, whether it was single or combination-based rehabilitation.&nbsp; Patient&nbsp; was&nbsp; assessed&nbsp; for progress&nbsp; achieved&nbsp; since&nbsp; the&nbsp; onset&nbsp; of stroke, functional status, balance and gait, cognition,&nbsp; family&nbsp; support&nbsp; and&nbsp; recently&nbsp; re- ceived&nbsp; interventions.&nbsp; History,&nbsp; physical&nbsp; ex- amination&nbsp; and&nbsp; standardized&nbsp; assessment tools&nbsp; were&nbsp; performed;&nbsp; patient&rsquo;s&nbsp; progress, strengths and weaknesses were discussed during&nbsp; the&nbsp; consultation.&nbsp; Based&nbsp; on&nbsp; the assessments, a set of goals that comprised functional and activities of daily living were generated and agreed upon by the patient or&nbsp; family&nbsp; members.&nbsp; A&nbsp; set&nbsp; of&nbsp; task&nbsp; specific activities comprised of physiotherapy inter-ventions&nbsp; and occupational therapy&nbsp; inter- ventions were planned for the patient. The occupational&nbsp; therapy team did baseline home&nbsp; assessments&nbsp; for patients who re- quired modifications to be done to facilitate their&nbsp; activities of living conditions. Speech and&nbsp; language as well as prosthetics and orthotics&nbsp; interventions were added if indicated.&nbsp;</p>\r\n<p>\r\n	Patients&nbsp; were seen on 3-monthly&nbsp; inter- vals in CSRC, in which progress or regres- sion of recovery&nbsp; was assessed. Based on the&nbsp; assessments,&nbsp; targeted goals were ei- ther intensified or reduced. Adjunctive ther- apies&nbsp; in the form&nbsp; of hydrotherapy,&nbsp; music therapy&nbsp; or counseling were added for&nbsp; patients who reached a plateau or regressed in&nbsp; their progress. For those who were ready&nbsp; to be discharged to the community, further interventions in the form of driving or back to work assessments, leisure or social rehabilitation&nbsp; intervention were planned. Throughout&nbsp; the two-year&nbsp; rehabilitation pe- riod,&nbsp; patients were structured into phases of&nbsp; intervention namely intensive individual therapy,&nbsp; guided individual therapy and group&nbsp; therapy; which were applicable for physiotherapy&nbsp; and occupational interven- tions. A summary of the CSRC program is as shown in Figure 1.</p>\r\n<p>\r\n	<em>Patients and methods&nbsp; </em></p>\r\n<p>\r\n	We followed all stroke patients (n=68) who were&nbsp; registered&nbsp; with&nbsp; the&nbsp; CSRC&nbsp; program since the start of the program in May 2008 for a period of two years till May 2010. We recorded&nbsp; demographic&nbsp; profiles,&nbsp; stroke profiles, types and episodes of intervention received&nbsp; whilst&nbsp; in&nbsp; the&nbsp; CSRC&nbsp; program&nbsp; and the&nbsp; outcome&nbsp; after&nbsp; a&nbsp; 2-year&nbsp; period&nbsp; of intervention&nbsp; or&nbsp; till&nbsp; discharged&nbsp; to&nbsp; the community&nbsp; whichever&nbsp; was&nbsp; earliest.&nbsp; The patients&nbsp; who&nbsp; defaulted&nbsp; or&nbsp; dropped&nbsp; out from&nbsp; the&nbsp; program&nbsp; were&nbsp; also&nbsp; recorded. Two&nbsp; assessment&nbsp; tools&nbsp; were&nbsp; used&nbsp; during the&nbsp; follow-up.&nbsp; Both&nbsp; assessment&nbsp; tools were used at baseline and three monthly intervals for a total period of 12 months:</p>\r\n<p class="rteindent1">\r\n	&bull;&nbsp; A&nbsp; Modified&nbsp; Barthel&nbsp; Index&nbsp; Score (MBI),&nbsp; (range&nbsp; 0&ndash;100)&nbsp; measures&nbsp; the degree&nbsp; of&nbsp; autonomy&nbsp; in&nbsp; daily&nbsp; living activities.&nbsp; The&nbsp; BI&nbsp; scores&nbsp; were&nbsp; cate- gorized&nbsp; as:&nbsp; less&nbsp; than&nbsp; 30&nbsp; as&nbsp; depend- ent and&nbsp; in&nbsp; need of maximum help in performing&nbsp; daily&nbsp; tasks;&nbsp; 30&ndash;70&nbsp; as semi-dependent&nbsp; and&nbsp; needed&nbsp; some degree of help; and more than 70 as independent&nbsp; in&nbsp; performing&nbsp; functional tasks (Wade &amp; Collin 1988).&nbsp;</p>\r\n<p class="rteindent1">\r\n	&bull;&nbsp; A Berg Balance Score (BBS), (range 0&ndash;56)&nbsp; measures&nbsp; impairment&nbsp; in&nbsp; bal- ance&nbsp; function&nbsp; by&nbsp; assessing&nbsp; the&nbsp; per- formance&nbsp; of&nbsp; functional&nbsp; tasks.&nbsp; The BBS&nbsp; was&nbsp; categorized&nbsp; as:&nbsp; scores&nbsp; of 0&ndash;20&nbsp; as&nbsp; having&nbsp; high&nbsp; fall&nbsp; risk;&nbsp; 21&ndash;40 as having medium fall risk and more than&nbsp; 40&nbsp; as&nbsp; having&nbsp; low&nbsp; fall&nbsp; risk&nbsp; (Berg et al. 1989).</p>\r\n<p>\r\n	<em>Statistical analysis&nbsp; </em></p>\r\n<p>\r\n	Data&nbsp; was&nbsp; analyzed&nbsp; using&nbsp; SPSS&nbsp; 14.&nbsp; De- scriptive&nbsp; analyses&nbsp; were&nbsp; used&nbsp; to&nbsp; present categorical data, in which results are pre- sented&nbsp; as&nbsp; mean+SD&nbsp; or&nbsp; proportions&nbsp; (%) as appropriate. The association between patients&rsquo;&nbsp; characteristics&nbsp; and&nbsp; outcomes after&nbsp; rehabilitation&nbsp; were&nbsp; assessed&nbsp; using multivariate&nbsp; analyses.&nbsp; A&nbsp; significant&nbsp; level of p&le;0.05 was set for the study.&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>RESULTS </strong></p>\r\n<p>\r\n	<em>Demographic characteristics&nbsp; </em></p>\r\n<p>\r\n	A total of 68 patients were recruited (the demographic characteristics are as Table 1). The mean age was 62.4 years (12.4) with the majority above 50 years old. The mean&nbsp; duration&nbsp; after&nbsp; the&nbsp; last&nbsp; stroke&nbsp; inci- dent&nbsp; was&nbsp; 21&nbsp; months&nbsp; (16.4).&nbsp; The&nbsp; mean post-stroke&nbsp; duration&nbsp; when&nbsp; first&nbsp; reviewed in&nbsp; CSRC&nbsp; was&nbsp; 11.5&nbsp; months&nbsp; (SD&nbsp; 11.9), with the earliest patient referred to CSRC after&nbsp; one&nbsp; month&nbsp; post-stroke&nbsp; and&nbsp; the&nbsp; lat- est 60 months post-stroke.</p>\r\n<p>\r\n	<em>Intervention&nbsp; received&nbsp; by&nbsp; patients&nbsp; in CSRC&nbsp; </em></p>\r\n<p>\r\n	Figure 2 and Table 1 illustrate types and outcome&nbsp; of&nbsp; rehabilitation&nbsp; intervention received&nbsp; by&nbsp; stroke&nbsp; patients&nbsp; after&nbsp; being reviewed&nbsp; in&nbsp; CSRC.&nbsp; Patients&nbsp; attending CSRC&nbsp; received&nbsp; interventions&nbsp; that&nbsp; varied from&nbsp; single&nbsp; to&nbsp; quadruple&nbsp; therapy;&nbsp; only 8.8%&nbsp; received&nbsp; single&nbsp; therapy&nbsp; during&nbsp; the intervention&nbsp; period.&nbsp; Outcomes&nbsp; of&nbsp; the&nbsp; patients&nbsp; demonstrated&nbsp; that&nbsp; majority&nbsp; of&nbsp; the patients&nbsp; were&nbsp; followed-up&nbsp; either&nbsp; by&nbsp; the primary&nbsp; care&nbsp; team&nbsp; specialized&nbsp; in&nbsp; long- term&nbsp; stroke&nbsp; care&nbsp; management&nbsp; or&nbsp; as&nbsp; a combination with other specialized clinics (neurology,&nbsp; psychiatry&nbsp; and&nbsp; orthopaedic clinics)&nbsp; based&nbsp; on&nbsp; the&nbsp; needs&nbsp; of&nbsp; the&nbsp; patients (n=25, 65.8%). The outcome of the patients&nbsp; showed&nbsp; that&nbsp; half&nbsp; of&nbsp; the&nbsp; patients were&nbsp; still&nbsp; in&nbsp; rehabilitation&nbsp; interventions, with only 17.6% who defaulted follow-up.</p>\r\n<p>\r\n	Scores&nbsp; from&nbsp; assessment&nbsp; tools&nbsp; demon- strated&nbsp; improvement&nbsp; over&nbsp; the&nbsp; twelve months&nbsp; (Table&nbsp; 2).&nbsp; Both&nbsp; assessment&nbsp; tools demonstrated&nbsp; an&nbsp; increasing&nbsp; trend throughout&nbsp; the&nbsp; follow-up&nbsp; period;&nbsp; MBI showed greater improvement. Multivariate analysis&nbsp; of&nbsp; progress&nbsp; over&nbsp; time&nbsp; for&nbsp; MBI demonstrated&nbsp; significant&nbsp; effect&nbsp; of&nbsp; inter- vention&nbsp; [F(3,7.0)=10.40,&nbsp; p=0.006].&nbsp; The BBS&nbsp; also&nbsp; demonstrated&nbsp; similar&nbsp; effect&nbsp; of the&nbsp; intervention&nbsp; over&nbsp; time&nbsp; [F(3,10)=5.53, p=0.017].&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION&nbsp; </strong></p>\r\n<p>\r\n	This&nbsp; prospective&nbsp; observational&nbsp; study&nbsp; in- vestigated&nbsp; the&nbsp; effectiveness&nbsp; of&nbsp; adding&nbsp; a multi-disciplinary&nbsp; structured&nbsp; review&nbsp; for outpatient&nbsp; stroke&nbsp; rehabilitation,&nbsp; which showed&nbsp; a&nbsp; significant&nbsp; improvement&nbsp; in terms&nbsp; of&nbsp; functional&nbsp; status&nbsp; and&nbsp; balance after a year of regular follow-up. In addition, three quarters of the stroke patients had&nbsp; continuity&nbsp; of&nbsp; care&nbsp; for&nbsp; their&nbsp; stroke problems&nbsp; in&nbsp; a&nbsp; designated&nbsp; primary&nbsp; care clinic&nbsp; and&nbsp; almost&nbsp; 40%&nbsp; were&nbsp; either&nbsp; dis- charged to the community or prepared to be&nbsp; discharged&nbsp; from&nbsp; intervention.&nbsp; These outcomes&nbsp; reflect&nbsp; the success&nbsp; of&nbsp; our combined&nbsp; stroke&nbsp; rehabilitation&nbsp; services, which&nbsp; aimed&nbsp; to solve&nbsp; the&nbsp; issues&nbsp; on how best&nbsp; to coordinate&nbsp; ongoing&nbsp; rehabilitation following&nbsp; discharge.&nbsp; The&nbsp; enrolment&nbsp; of patients&nbsp; was&nbsp; not guided&nbsp; by&nbsp; any&nbsp; criteria, and&nbsp; depended&nbsp; only&nbsp; on patients&rsquo;&nbsp; referral from individual rehabilitation units or from the wards they had been discharged.&nbsp; As the&nbsp; patients&nbsp; recruited&nbsp; for&nbsp; this&nbsp; study&nbsp; were independent&nbsp; of&nbsp; any&nbsp; characteristics,&nbsp; be&nbsp; it severity&nbsp; or&nbsp; potential&nbsp; to&nbsp; improve,&nbsp; out- comes&nbsp; demonstrated&nbsp; the true&nbsp; picture&nbsp; of stroke patients in the community.</p>\r\n<p>\r\n	The program we used&nbsp; in this study fol- lows&nbsp; the consensus&nbsp; that&nbsp; recovery&nbsp; after stroke&nbsp; might&nbsp; progress&nbsp; beyond&nbsp; plateau phase,&nbsp; as&nbsp; suggested&nbsp; by&nbsp; recent&nbsp; neuro- imaging&nbsp; studies&nbsp; (Seitz&nbsp; et&nbsp; al.&nbsp; 1995;&nbsp; Jo- hansson&nbsp; 2000).&nbsp; Our&nbsp; observations&nbsp; dem- onstrated&nbsp; an increased&nbsp; trend&nbsp; in&nbsp; both Modified Barthel Index and Berg Balance Scale&nbsp; scores&nbsp; over&nbsp; twelve&nbsp; months&nbsp; follow- up&nbsp; in&nbsp; CSRC.&nbsp; Despite&nbsp; the&nbsp; mean&nbsp; stroke duration&nbsp; first&nbsp; seen&nbsp; in&nbsp; CSRC&nbsp; was&nbsp; quite late&nbsp; (11.5&nbsp; months),&nbsp; the&nbsp; results&nbsp; suggest that patients post-stroke were able to im- prove in functional recovery if continuous and&nbsp; structured&nbsp; rehabilitation&nbsp; addressed the&nbsp; real&nbsp; problems&nbsp; and&nbsp; needs&nbsp; of&nbsp; the&nbsp; pa- tients,&nbsp; rather&nbsp; than&nbsp; standard&nbsp; therapy&nbsp; that most&nbsp; stroke&nbsp; survivors&nbsp; now&nbsp; receive.&nbsp; Our approach&nbsp; in&nbsp; combining&nbsp; more&nbsp; than&nbsp; one therapy&nbsp; intervention&nbsp; in&nbsp; a&nbsp; single&nbsp; day&nbsp; may accentuate&nbsp; the&nbsp; effect&nbsp; of&nbsp; the&nbsp; intervention to&nbsp; the&nbsp; survivors,&nbsp; hence&nbsp; the&nbsp; improved functional&nbsp; recovery&nbsp; over-time.&nbsp;&nbsp;&nbsp; However, a&nbsp; randomized&nbsp; controlled&nbsp; trial&nbsp; comparing this&nbsp; new&nbsp; intervention&nbsp; with&nbsp; the&nbsp; standard approach may be able to substantiate the effectiveness of this approach.</p>\r\n<p>\r\n	A&nbsp; comparison&nbsp; of&nbsp; our&nbsp; program&nbsp; to those of&nbsp; post-discharge&nbsp; stroke&nbsp; interventions (Kuptniratsaikul&nbsp; et al. 2009; Grasel et al. 2006;&nbsp; Hartman-Maeir&nbsp; et al.&nbsp; 2007)&nbsp; dem- onstrated a similar trend in the long-term outcome of stroke patients. Kapniratsakul and&nbsp; Grasel&nbsp; provided&nbsp; intervention&nbsp; whilst the&nbsp; patients&nbsp; were&nbsp; in the&nbsp; ward,&nbsp; whereas Maeir continued the intervention following discharge&nbsp; to&nbsp; the&nbsp; community.&nbsp; All&nbsp; these findings demonstrated an improvement in terms of functional outcomes, awareness to&nbsp; health&nbsp; facilities&nbsp; and&nbsp; level&nbsp; of&nbsp; activities that&nbsp; were&nbsp; consistent&nbsp; with&nbsp; our&nbsp; current findings. Our hypothesis is that the provi- sion of continuity of care and multidiscip- linary team care approach has the poten- tial to emulate the success of acute care management of stroke patients, in&nbsp; which involvement&nbsp; of&nbsp; multiple&nbsp; disciplines&nbsp; with regular&nbsp; meetings&nbsp; and&nbsp; assessments&nbsp; im- proved&nbsp; both&nbsp; survival&nbsp; and&nbsp; functional&nbsp; out- comes&nbsp; over&nbsp; long-term follow-up&nbsp; (Kalra&nbsp; &amp; Langhorne 2007; Rodgers et al. 1999).</p>\r\n<p>\r\n	Although&nbsp; it&nbsp; is&nbsp; evident&nbsp; that&nbsp; there&nbsp; is&nbsp; an apparent&nbsp; shift&nbsp; towards&nbsp; providing&nbsp; a&nbsp; multi- disciplinary&nbsp; rehabilitation&nbsp; to&nbsp; post-dis- charge stroke patients in the community, variability was apparent in terms of provi- sion&nbsp; of&nbsp; care&nbsp; in&nbsp; previous&nbsp; studies&nbsp; and&nbsp; of the current program. Our program, which was&nbsp; outpatient-based,&nbsp; was&nbsp; successful due to the fact that it was held as an ex- tension&nbsp; of&nbsp; the&nbsp; existing&nbsp; stroke&nbsp; rehabilita- tion&nbsp; service&nbsp; in&nbsp; the&nbsp; hospital.&nbsp; Although&nbsp; the rehabilitation&nbsp; intervention&nbsp; program&nbsp; for stroke patients was already in place prior to&nbsp; CSRC,&nbsp; there&nbsp; were&nbsp; no&nbsp; structured&nbsp; as- sessments&nbsp; or&nbsp; evaluations&nbsp; performed during&nbsp; the&nbsp; routine&nbsp; two&nbsp; years&nbsp; period&nbsp; of intervention.&nbsp; Hence,&nbsp; the&nbsp; CSRC&nbsp; provided a&nbsp; one&nbsp; stop-point&nbsp; for&nbsp; clinicians&nbsp; and&nbsp; the- rapists&nbsp; to&nbsp; discuss&nbsp; and&nbsp; individualize&nbsp; the treatment&nbsp; plan&nbsp; for&nbsp; each&nbsp; patient.&nbsp; This&nbsp; in turn,&nbsp; enabled&nbsp; us to prognosticate&nbsp; the re- covery for patients thus giving them rea- listic expectations in their recovery.&nbsp;</p>\r\n<p>\r\n	In&nbsp; view&nbsp; of&nbsp; the&nbsp; optimum&nbsp; place&nbsp; for&nbsp; post- stroke&nbsp; rehabilitation,&nbsp; inpatient&nbsp; rehabilita- tion&nbsp; has&nbsp; been&nbsp; acknowledged&nbsp; to&nbsp; provide the&nbsp; most&nbsp; intensive&nbsp; rehabilitation&nbsp; for&nbsp; pa- tients&nbsp; with&nbsp; variable&nbsp; level&nbsp; of&nbsp; disabilities; however&nbsp; the&nbsp; choice&nbsp; of&nbsp; rehabilitation should be determined not only by the pa- tient&nbsp; needs,&nbsp; but&nbsp; should&nbsp; consider&nbsp; other factors&nbsp; as&nbsp; well&nbsp; (Lee&nbsp; et&nbsp; al.&nbsp; 1997).&nbsp; In&nbsp; this program, the decision of providing follow- through&nbsp; rehabilitation&nbsp; intervention&nbsp; as&nbsp; an outpatient&nbsp; basis&nbsp; was&nbsp; based&nbsp; on&nbsp; several factors&nbsp; namely&nbsp; familiarity&nbsp; of&nbsp; post-stroke patients to the hospital set-up, post-acute care availability and most importantly the aims&nbsp; of&nbsp; providing&nbsp; continuous&nbsp; rehabilita- tion&nbsp; to these&nbsp; patients.&nbsp; The&nbsp; early&nbsp; findings of our results suggest out-patient rehabil- itation&nbsp; program&nbsp; with a multi-disciplinary approach&nbsp; might&nbsp; be the&nbsp; catalyst&nbsp; for&nbsp; a more&nbsp; comprehensive&nbsp; care&nbsp; of&nbsp; stroke&nbsp; pa- tients in the community, with the rehabil- itation providing the link to other services at this stage of recovery.</p>\r\n<p>\r\n	There&nbsp; are&nbsp; several&nbsp; limitations&nbsp; to&nbsp; the study.&nbsp; Since&nbsp; the&nbsp; study&nbsp; was&nbsp; an observa- tional&nbsp; study,&nbsp; no&nbsp; comparison&nbsp; was&nbsp; made with patients not with the CSRC program. Further&nbsp; research&nbsp; should&nbsp; be done to in- vestigate&nbsp; the&nbsp; effectiveness&nbsp; of&nbsp; this&nbsp; pro- gram&nbsp; using&nbsp; a larger&nbsp; sample&nbsp; size&nbsp; for&nbsp; it to be recommended as standard practice.</p>\r\n<p>\r\n	In&nbsp; conclusion,&nbsp; the&nbsp; CSRC&nbsp; provides&nbsp; a structured&nbsp; rehabilitation&nbsp; intervention&nbsp; that was&nbsp; beneficial&nbsp; to&nbsp; post-stroke&nbsp; patients&nbsp; in terms&nbsp; of&nbsp; functional&nbsp; status,&nbsp; and&nbsp; improve- ment&nbsp; in&nbsp; balance&nbsp; and&nbsp; mobility.&nbsp; It&nbsp; also showed that stroke patients over a period of six months and beyond are able to im- prove if given sufficient intervention.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS </strong></p>\r\n<p>\r\n	&nbsp;The&nbsp; authors&nbsp; wish&nbsp; to&nbsp; thank&nbsp; the&nbsp; stoke&nbsp; pa- tients&nbsp; and&nbsp; their&nbsp; families&nbsp; for&nbsp; their&nbsp; support and cooperation in this new project. This CSRC project is part of the overall initia- tive of UKMMC to facilitate a comprehen- sive&nbsp; pathway&nbsp; for&nbsp; stroke&nbsp; care&nbsp; from&nbsp; acute care,&nbsp; intervention,&nbsp; rehabilitation&nbsp; and community&nbsp; care&nbsp; under&nbsp; the&nbsp; flagship&nbsp; of Kuala&nbsp; Lumpur&nbsp; Regional&nbsp; Interventional Stroke Strategy System (KRISIS) project.&nbsp;&nbsp;&nbsp;</p>\r\n', 2),
(471, 468, '<p>\r\n	<strong>&nbsp;INTRODUCTION&nbsp; </strong></p>\r\n<p>\r\n	Amitraz&nbsp; is&nbsp; a&nbsp; synthetic&nbsp; compound,&nbsp; widely used&nbsp; in&nbsp; veterinary&nbsp; medicine&nbsp; as&nbsp; an insecticide&nbsp; and&nbsp; acaricide.&nbsp; The&nbsp; IUPAC name&nbsp; is&nbsp; 1,5&nbsp; di-(2,&nbsp; 4-dimethylphenyl)-3- methyl-1,3,&nbsp; 5-triaza-penta-1,&nbsp; 4-diene,&nbsp; a member&nbsp; of&nbsp; the&nbsp; formamidine&nbsp; pesticide group. It is generally used as a treatment for&nbsp; control&nbsp; of&nbsp; ticks&nbsp; and&nbsp; mites&nbsp; in&nbsp; dogs (Shitole&nbsp; et&nbsp; al.&nbsp; 2010).&nbsp; Only&nbsp; a&nbsp; handful&nbsp; of Amitraz&nbsp; poisoning&nbsp; cases&nbsp; are&nbsp; reported&nbsp; in the literature and to date, no such cases are described in the Malaysian literature. We&nbsp; report&nbsp; a&nbsp; case&nbsp; of&nbsp; amitraz&nbsp; poisoning and&nbsp; review&nbsp; the&nbsp; clinical&nbsp; features&nbsp; and management of his condition.&nbsp;</p>\r\n<p>\r\n	<strong>CASE REPORT&nbsp; </strong></p>\r\n<p>\r\n	A&nbsp; healthy&nbsp; 18&nbsp; year-old&nbsp; man&nbsp; had&nbsp; ingested an approximately 100ml of undiluted anti flea&nbsp; shampoo&nbsp; for&nbsp; dogs&nbsp; in&nbsp; a&nbsp; suicidal attempt.&nbsp; He&nbsp; was&nbsp; brought&nbsp; into&nbsp; the Emergency&nbsp; Department&nbsp; an&nbsp; hour&nbsp; after ingestion with an initial blood pressure of 115/60&nbsp; mmHg&nbsp; and&nbsp; a&nbsp; pulse&nbsp; rate&nbsp; of&nbsp; 110 beats&nbsp; per&nbsp; minute.&nbsp; He&nbsp; was&nbsp; alert&nbsp; with&nbsp; a Glasgow&nbsp; Coma&nbsp; Scale&nbsp; (GCS)&nbsp; score&nbsp; of 15/15&nbsp; and&nbsp; a&nbsp; blood&nbsp; capillary&nbsp; glucose&nbsp; of 5.4&nbsp; mmol/L.&nbsp; However,&nbsp; he&nbsp; was&nbsp; noted&nbsp; to be drowsy an hour after admission with a reduced&nbsp; GCS&nbsp; score&nbsp; of&nbsp; 8/15.&nbsp; His&nbsp; blood pressure also plummeted to 75/40 mmHg with a pulse rate of 60 beats per minute. Accordingly, he&nbsp; was intubated for airway protection&nbsp; and&nbsp; started&nbsp; on&nbsp; inotropic support&nbsp; (noradrenaline).&nbsp; Blood&nbsp; tests revealed&nbsp; normal&nbsp; renal&nbsp; and&nbsp; liver&nbsp; function. He&nbsp; was&nbsp; normoglycemic&nbsp; throughout. Toxicology screen for other poisons were negative.</p>\r\n<p>\r\n	After&nbsp; inotropic&nbsp; support&nbsp; was&nbsp; tapered&nbsp; off within&nbsp; 8&nbsp; hours,&nbsp; his&nbsp; condition&nbsp; improved over&nbsp; the&nbsp; next&nbsp; three&nbsp; days&nbsp; and&nbsp; was subsequently discharged well.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION&nbsp; </strong></p>\r\n<p>\r\n	Amitraz is a potent &alpha;2-adrenergic agonist. Stimulation&nbsp; of&nbsp; central&nbsp; &alpha;2-adrenergic receptors&nbsp; results&nbsp; in&nbsp; hypotension&nbsp; and bradycardia&nbsp; (Jones&nbsp; 1990).&nbsp; Common clinical&nbsp; presentations&nbsp; include&nbsp; central nervous&nbsp; depression&nbsp; ranging&nbsp; from drowsiness to coma (Bonsall et al. 1983). Other&nbsp; reported&nbsp; effects&nbsp; of&nbsp; Amitraz poisoning&nbsp; are&nbsp; hyperglycemia, hypothermia&nbsp; and&nbsp; convulsions&nbsp; (Leung&nbsp; et al. 1999). Organophosphates, opiods and drugs&nbsp; that&nbsp; act&nbsp; at&nbsp; &alpha;2-adrenergic receptors such&nbsp; as&nbsp; clinodine&nbsp; can&nbsp; also&nbsp; present similarly&nbsp; and&nbsp; might&nbsp; lead&nbsp; to&nbsp; diagnostic confusion.&nbsp; The&nbsp; reported&nbsp; time&nbsp; from ingestion&nbsp; to&nbsp; the&nbsp; onset&nbsp; of&nbsp; signs&nbsp; and symptoms is about 30 to 90 minutes. The duration&nbsp; of&nbsp; recovery&nbsp; varied&nbsp; from&nbsp; 2&nbsp; to&nbsp; 48 hours after presentation (Avşaroğullari et al.&nbsp; 2006).&nbsp; Our&nbsp; patient&nbsp; had&nbsp; central nervous depression with hypotension and bradycardia&nbsp; two&nbsp; hours&nbsp; after&nbsp; ingestion which&nbsp; was&nbsp; consistent&nbsp; with&nbsp; Amitraz&rsquo;s effects.&nbsp; He&nbsp; had&nbsp; a&nbsp; relatively&nbsp; quick recovery&nbsp; and&nbsp; his&nbsp; condition&nbsp; improved&nbsp; a day&nbsp; after&nbsp; ingestion.&nbsp; However,&nbsp; there&nbsp; was neither hyperglycemia nor hypothermia in him.&nbsp; Clinical&nbsp; features&nbsp; such&nbsp; as unconsciousness,&nbsp; hypotension&nbsp; and bradycardia&nbsp; requiring&nbsp; mechanical ventilation&nbsp; were&nbsp; described&nbsp; in&nbsp; similar cases&nbsp; (Shitole&nbsp; et&nbsp; al.&nbsp; 2010).&nbsp; Yilmaz&nbsp; et&nbsp; al. has&nbsp; also&nbsp; described&nbsp; severe&nbsp; respiratory depression&nbsp; requiring&nbsp; mechanical ventilation for less than 24 hours.&nbsp;</p>\r\n<p>\r\n	Treatment&nbsp; of&nbsp; Amitraz&nbsp; poisoning&nbsp; is mainly&nbsp; supportive&nbsp; and&nbsp; symptomatic. There&nbsp; is&nbsp; no&nbsp; specific&nbsp; antidote&nbsp; for&nbsp; this poisoning&nbsp; (Yilmaz&nbsp; et&nbsp; al.&nbsp; 2003).&nbsp; In&nbsp; some animal studies, &alpha;2-adrenergic antagonists such&nbsp; as&nbsp; yohimbin&nbsp; and&nbsp; atimepazole reversed&nbsp; most&nbsp; of&nbsp; the&nbsp; signs&nbsp; in&nbsp; Amitraz poisoning.&nbsp; However,&nbsp; these&nbsp; antagonists have&nbsp; never&nbsp; been&nbsp; used&nbsp; in&nbsp; humans&nbsp; and should&nbsp; only&nbsp; be&nbsp; considered&nbsp; in&nbsp; severe cases&nbsp; (Avşaroğullari&nbsp; et&nbsp; al.&nbsp; 2006).&nbsp; The main&nbsp; goal&nbsp; of&nbsp; management&nbsp; in&nbsp; Amitraz poisoning&nbsp; is&nbsp; airway&nbsp; clearance&nbsp; with adequate ventilation in cases of impaired consciousness.&nbsp; Inotropic&nbsp; agents (dopamine&nbsp; and&nbsp; noradrenaline)&nbsp; may&nbsp; be required in hypotensive patients who are not&nbsp; responsive&nbsp; to&nbsp; fluid&nbsp; challenges (Avşaroğullari&nbsp; et&nbsp; al.&nbsp; 2006).&nbsp; Atropine&nbsp; may be&nbsp; useful&nbsp; for&nbsp; treating&nbsp; hemodynamically unstable&nbsp; bradycardia&nbsp; (Jones&nbsp; 1990). Despite a life threatening&nbsp; clinical picture, Amitraz&nbsp; poisoning&nbsp; in&nbsp; humans&nbsp; carries&nbsp; a low&nbsp; mortality&nbsp; when&nbsp; appropriate supportive&nbsp; therapy&nbsp; is&nbsp; given&nbsp; (Shitole 2010).</p>\r\n', 1),
(472, 469, '<p>\r\n	<strong>INTRODUCTION&nbsp;&nbsp; </strong></p>\r\n<p>\r\n	Coagulation&nbsp; tests&nbsp; are&nbsp; usually&nbsp; requested by&nbsp; clinicians&nbsp; for&nbsp; a&nbsp; variety&nbsp; of&nbsp; reasons.&nbsp; It could be part of an investigation for a pa- tient&nbsp; with&nbsp; bleeding&nbsp; tendencies&nbsp; or&nbsp; a&nbsp; rou- tine&nbsp; test&nbsp; prior&nbsp; to&nbsp; a&nbsp; surgical&nbsp; procedure. With&nbsp; the&nbsp; increasing&nbsp; trend&nbsp; of&nbsp; routine&nbsp; coa- gulation&nbsp; testing,&nbsp; the&nbsp; haemostasis&nbsp; laboratory&nbsp; is&nbsp; often&nbsp; burdened&nbsp; with&nbsp; the&nbsp; task&nbsp; of investigating&nbsp; an&nbsp; isolated&nbsp; prolonged APTT. In most cases, these turned out to be&nbsp; inconclusive&nbsp; or&nbsp; due&nbsp; to&nbsp; factor&nbsp; XII&nbsp; defi- ciency which is of no clinical significance. In this case report&nbsp; we illustrate&nbsp; a patient with&nbsp; chronic&nbsp; myelomonocytic&nbsp; leukaemia who&nbsp; presented&nbsp; with&nbsp; multiple&nbsp; subcutane- ous&nbsp; abscesses.&nbsp; The&nbsp; initial&nbsp; investigation showed&nbsp; a&nbsp; prolonged&nbsp; APTT,&nbsp; a&nbsp; low&nbsp; factor IX level and the presence of an inhibitor. She was diagnosed as an acquired factor IX inhibitor. On further testing this turned out&nbsp; to&nbsp; be&nbsp; a&nbsp; lupus&nbsp; anticoagulant. We&nbsp; dis- cuss&nbsp; the&nbsp; investigation&nbsp; of&nbsp; a&nbsp; prolonged APTT&nbsp; and&nbsp; how&nbsp; to&nbsp; differentiate&nbsp; a&nbsp; specific factor&nbsp; inhibitor&nbsp; from&nbsp; a&nbsp; lupus&nbsp; anticoagu- lant. This is the first case report that de- monstrates correction of an apparent low factor&nbsp; level&nbsp; with&nbsp; phopholipid&nbsp; mixing&nbsp; studies&nbsp; in&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; lupus&nbsp; anticoa- gulant.&nbsp;</p>\r\n<p>\r\n	<strong>CASE REPORT </strong></p>\r\n<p>\r\n	&nbsp;A&nbsp; 51&nbsp; year-old&nbsp; lady&nbsp; was&nbsp; diagnosed&nbsp; with chronic&nbsp; myelomonocytic&nbsp; leukaemia&nbsp; in August&nbsp; 2004&nbsp; when&nbsp; she&nbsp; presented&nbsp; with epigastric pain and was noted to have an enlarged liver and spleen with monocyto- sis.&nbsp; She&nbsp; remained&nbsp; well&nbsp; following&nbsp; treatment with low dose cytarabine until three years later when she presented with multiple&nbsp; large&nbsp; abscesses&nbsp; over&nbsp; the buttocks, thigh and abdomen. She was seen by the orthopaedic surgeon and was planned for drainage&nbsp; and&nbsp; saucerisation&nbsp; of&nbsp; the ab- scesses.&nbsp; She had no bleeding&nbsp; symp- toms.&nbsp; Laboratory&nbsp; results&nbsp; included&nbsp; a hae- moglobin&nbsp; of&nbsp; 10.4g/dL,&nbsp; a white&nbsp; blood&nbsp; cell count&nbsp; of&nbsp; 5.6x109/L,&nbsp; and&nbsp; a platelet&nbsp; count of&nbsp; 41x109/L.&nbsp; Prothrombin&nbsp; time&nbsp; was&nbsp; 12.2 seconds&nbsp; (normal,&nbsp; 10.2-13.2&nbsp; sec);&nbsp; APTT, 72.2&nbsp; seconds&nbsp; (normal,&nbsp; 21-32&nbsp; sec)&nbsp; and fibrinogen,&nbsp; 362&nbsp; mg/dL&nbsp; (normal&nbsp; 150-450 mg/dL). Factor VIII:C activity was 72.9%; factor IX:C activity&nbsp; was 4.6%, with factor IX inhibitor of 4.0 Bethesda units (BU). In view&nbsp; of&nbsp; the results,&nbsp; she&nbsp; was&nbsp; diagnosed as having an acquired factor IX inhibitor. She&nbsp; was&nbsp; treated&nbsp; with&nbsp; recombinant&nbsp; acti- vated&nbsp; factor&nbsp; VII at 90mcg/kg&nbsp; and under- went the surgery without any bleeding or thrombotic&nbsp; complications.&nbsp; She was&nbsp; also given&nbsp; a course&nbsp; of&nbsp; antibiotics.&nbsp; However, three&nbsp; weeks&nbsp; later&nbsp; she&nbsp; returned&nbsp; with&nbsp; re- currence&nbsp; of&nbsp; the&nbsp; abscesses&nbsp; in&nbsp; the&nbsp; left axilla and back.</p>\r\n<p>\r\n	The&nbsp; patient&nbsp; was&nbsp; now&nbsp; referred&nbsp; to&nbsp; our hematology&nbsp; department&nbsp; for&nbsp; further&nbsp; man- agement.&nbsp; She&nbsp; had&nbsp; no&nbsp; bleeding symptoms. A repeat coagulation test was done.&nbsp; The&nbsp; results&nbsp; showed&nbsp; PT&nbsp; was&nbsp; 14.7 seconds&nbsp; (normal,&nbsp; 11.0-14.5&nbsp; sec),&nbsp; APTT was&nbsp; 120.0&nbsp; seconds&nbsp; (normal,&nbsp; 28-40 seconds)&nbsp; and&nbsp; the&nbsp; mixing&nbsp; studies&nbsp; with normal&nbsp; plasma&nbsp; showed&nbsp; a&nbsp; persistent prolongation of the APTT at 96.5 seconds suggesting&nbsp; the&nbsp; presence&nbsp; of&nbsp; an&nbsp; inhibitor. Factor&nbsp; VIII:C&nbsp; was&nbsp; 22%&nbsp; (normal,&nbsp; 60%- 150%),&nbsp; factor&nbsp; IX:C&nbsp; was&nbsp; &lt;1.0%&nbsp; (normal, 60%-150%),&nbsp; factor&nbsp; XI&nbsp; was&nbsp; &lt;1.0%&nbsp; and factor&nbsp; XII&nbsp; &lt;1.0%.&nbsp; Serial&nbsp; dilutions&nbsp; for&nbsp; fac-tor levels were assayed to look for speci- ficity of the inhibitor (Table 1). A chromogenic&nbsp; factor&nbsp; VIII&nbsp; assay&nbsp; was&nbsp; also&nbsp; done and&nbsp; this&nbsp; showed&nbsp; a&nbsp; factor&nbsp; VIII&nbsp; level&nbsp; of 120%. To demonstrate that the low factor levels&nbsp; were&nbsp; due&nbsp; to&nbsp; the&nbsp; presence&nbsp; of&nbsp; a lupus&nbsp; anticoagulant&nbsp; (LA),&nbsp; phospholipid was&nbsp; added&nbsp; to&nbsp; the&nbsp; factor&nbsp; assays.&nbsp; The factor&nbsp; levels&nbsp; rose&nbsp; confirming&nbsp; that&nbsp; the apparent&nbsp; low&nbsp; factor&nbsp; levels&nbsp; were&nbsp; due&nbsp; to the interference by the LA (Table 2). The platelet&nbsp; count&nbsp; was&nbsp; 80x109/L&nbsp; with&nbsp; the presence&nbsp; of&nbsp; giant&nbsp; platelets&nbsp; and&nbsp; platelet clumps.&nbsp; Further&nbsp; testing&nbsp; for&nbsp; LA&nbsp; was carried&nbsp; out.&nbsp; The&nbsp; dilute&nbsp; Russell&rsquo;s&nbsp; viper venom&nbsp; test&nbsp; (DRVVT)&nbsp; was&nbsp; prolonged&nbsp; at 88.2&nbsp; seconds&nbsp; (normal&nbsp; range&nbsp; 26-36 seconds)&nbsp; and&nbsp; corrected&nbsp; to&nbsp; 42.3&nbsp; seconds with the addition of phospholipid, giving a normalized&nbsp; DRVVT&nbsp; ratio&nbsp; of&nbsp; 2.6&nbsp; (normal range&nbsp; up&nbsp; to&nbsp; 1.2)&nbsp; (Table&nbsp; 3).&nbsp; The hexagonal&nbsp; II&nbsp; phase&nbsp; phosphatidyle- thanolamine&nbsp; assay&nbsp; (Staclot-LA),&nbsp; showed a positive test for LA at 54 seconds (neg- ative &le; 8 sec) (Table 4). The diagnosis of acquired factor IX inhibitor was therefore revised&nbsp; and&nbsp; the&nbsp; prolonged&nbsp; APTT&nbsp; was attributed&nbsp; to&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; LA.&nbsp; The patient&nbsp; underwent&nbsp; a&nbsp; second&nbsp; surgical drainage&nbsp; and&nbsp; saucerisation&nbsp; of&nbsp; the&nbsp; ab- scesses,&nbsp; this&nbsp; time&nbsp; without&nbsp; any&nbsp; factor cover.&nbsp; There&nbsp; was&nbsp; no&nbsp; bleeding&nbsp; complica- tion.&nbsp; Her&nbsp; blood&nbsp; cultures&nbsp; were&nbsp; negative but&nbsp; the&nbsp; indirect&nbsp; fluorescent&nbsp; antibody&nbsp; titer for&nbsp; meloidosis&nbsp; was&nbsp; positive&nbsp; (1:160).&nbsp; She responded&nbsp; to&nbsp; treatment&nbsp; with&nbsp; intravenous ceftazidime and oral cotrimoxazole.&nbsp; She has had no recurrence of the abscesses and&nbsp; is&nbsp; on&nbsp; long-term&nbsp; treatment&nbsp; with&nbsp; cotrimoxazole. A repeat test 6 months later showed persistence of the LA.</p>\r\n<p>\r\n	<strong>MATERIALS AND METHODS&nbsp; </strong></p>\r\n<p>\r\n	The&nbsp; PT,&nbsp; APTT&nbsp; and&nbsp; factor&nbsp; assays&nbsp; were determined&nbsp; by&nbsp; using&nbsp; an&nbsp; automated&nbsp; coa- gulation&nbsp; analyzer&nbsp; (ACL&nbsp; 9000,&nbsp; Instrument Laboratory,&nbsp; USA).&nbsp; The&nbsp; PT&nbsp; reagents&nbsp; in- clude&nbsp; fibrinogen&nbsp; HS&nbsp; and&nbsp; calcium&nbsp; throm- boplastin (Rabbit-brain tissue), the APTT reagents&nbsp; consist&nbsp; of&nbsp; phospholipids&nbsp; (syn- thetic&nbsp; derived&nbsp; for&nbsp; optimal&nbsp; platelet&nbsp; lite&nbsp; ac- tivity)&nbsp; and&nbsp; calcium&nbsp; chloride&nbsp; (0.020M).&nbsp; All these&nbsp; reagents&nbsp; were&nbsp; supplied&nbsp; by&nbsp; the company from the Instrument Laboratory Sdn. Bhd. Mixing studies were performed using&nbsp; pooled&nbsp; plasma&nbsp; from&nbsp; 50&nbsp; healthy normal&nbsp; individuals&nbsp; recruited&nbsp; from&nbsp; our hospital. The factor deficient plasma was commercially&nbsp; obtained&nbsp; from&nbsp; the Instru- ment Laboratory (USA). The presence of LA&nbsp; was&nbsp; detected&nbsp; by&nbsp; the dilute&nbsp; Russell&rsquo;s viper venom test (DRVVT) and the Staclot LA. The phospholipid from the Staclot LA was&nbsp; used&nbsp; in&nbsp; the&nbsp; phospholipid&nbsp; mixing studies.&nbsp;</p>\r\n<p>\r\n	<strong>DISCUSSION </strong></p>\r\n<p>\r\n	The activated partial thromboplastin time (APTT) is a non-specific screening test of the intrinsic system while the prothrombin time&nbsp; (PT)&nbsp; is&nbsp; a&nbsp; screening&nbsp; test&nbsp; of&nbsp; the&nbsp; ex- trinsic&nbsp; system.&nbsp; A&nbsp; prolonged&nbsp; APTT&nbsp; with&nbsp; a normal PT indicates a possible deficiency of&nbsp; factors&nbsp; VIII,&nbsp; IX,&nbsp; XI&nbsp; or&nbsp; XII&nbsp; or&nbsp; the&nbsp; pres- ence&nbsp; of&nbsp; inhibitors.&nbsp; These&nbsp; inhibitors&nbsp; are either specific to these factors or are non- specific,&nbsp; the&nbsp; most&nbsp; common&nbsp; of&nbsp; which&nbsp; is the lupus anticoagulant (LA) (Chng et al. 2005).&nbsp; No&nbsp; single&nbsp; test&nbsp; is&nbsp; definitive&nbsp; for&nbsp; a LA. As a result, a variety of tests and also the clinical history are used in an attempt to&nbsp; establish&nbsp; the&nbsp; diagnosis&nbsp; (Lupus&nbsp; anti- coagulant&nbsp; working&nbsp; party&nbsp; 1991).&nbsp; In&nbsp; the present case, a misdiagnosis of factor IX inhibitor&nbsp; was&nbsp; made&nbsp; based&nbsp; on&nbsp; the&nbsp; initial test&nbsp; showing&nbsp; a&nbsp; low&nbsp; factor&nbsp; IX&nbsp; with&nbsp; normal factor&nbsp; VIII&nbsp; levels.&nbsp; The&nbsp; clinical&nbsp; history&nbsp; is very&nbsp; important&nbsp; to&nbsp; help&nbsp; us&nbsp; decide&nbsp; what further tests need to be done.&nbsp; A specific factor&nbsp; inhibitor&nbsp; usually&nbsp; has&nbsp; bleeding&nbsp; ma- nifestations.&nbsp; In&nbsp; this&nbsp; case&nbsp; the&nbsp; patient&nbsp; had no&nbsp; bleeding&nbsp; symptoms,&nbsp; so&nbsp; one&nbsp; should suspect the presence of a LA. Lupus anticoagulant&nbsp; can&nbsp; affect&nbsp; the apparent&nbsp; level of&nbsp; coagulation&nbsp; factors&nbsp; by&nbsp; interfering&nbsp; with the&nbsp; phospholipid-dependent&nbsp; one-stage assay based on APTT. In our experience factor IX and XI assays are affected mar- kedly&nbsp; than&nbsp; factor&nbsp; VIII.&nbsp; To&nbsp; overcome&nbsp; this, dilution&nbsp; assays&nbsp; on the&nbsp; patient&rsquo;s&nbsp; plasma should have been carried out to dilute out the&nbsp; effect&nbsp; of&nbsp; the lupus&nbsp; inhibitor&nbsp; (Kasper 1991).&nbsp;</p>\r\n<p>\r\n	In&nbsp; our&nbsp; laboratory,&nbsp; we found&nbsp; multiple factor&nbsp; deficiencies&nbsp; which&nbsp; made&nbsp; it easier as&nbsp; we&nbsp; then&nbsp; knew&nbsp; we were&nbsp; most&nbsp; likely dealing&nbsp; with&nbsp; a nonspecific&nbsp; inhibitor&nbsp; such as a LA. The dilution assays however did not&nbsp; show&nbsp; a rise&nbsp; in&nbsp; the&nbsp; apparent&nbsp; factor VIII, IX, XI and XII levels as would be ex- pected in the presence of a LA with factor levels remaining low in the presence of a specific&nbsp; inhibitor&nbsp; (Kasper&nbsp; 1991).&nbsp; This could be explained by the interference of a&nbsp; very&nbsp; strong&nbsp; lupus&nbsp; inhibitor.&nbsp; The&nbsp; factor VIII level did rise a little but fell to &lt; 1.0% at dilutions above 1/50, most likely due to dilution&nbsp; of&nbsp; the factor&nbsp; itself.&nbsp; To&nbsp; further prove that&nbsp; the apparent&nbsp; low factor levels were&nbsp; due to the&nbsp; presence&nbsp; of&nbsp; the LA, we added&nbsp; phospholipid&nbsp; to the patient&rsquo;s plasma&nbsp; and repeated&nbsp; the&nbsp; factor&nbsp; assays. Addition&nbsp; of&nbsp; phospholipid&nbsp; neutralizes&nbsp; the effect&nbsp; of&nbsp; LA and&nbsp; will&nbsp; correct&nbsp; the&nbsp; clotting times&nbsp; in the APTT-based&nbsp; factor&nbsp; assays (Tripodi 2007). This was clearly shown by the&nbsp; normalization&nbsp; of&nbsp; the apparent&nbsp; low factor&nbsp; levels&nbsp; with&nbsp; addition&nbsp; of&nbsp; phospholi- pid.&nbsp; It should&nbsp; be noted&nbsp; that&nbsp; addition&nbsp; of phospholipid&nbsp; to normal&nbsp; plasma&nbsp; produces a small dilution effect reducing the factor level&nbsp; slightly.&nbsp; We&nbsp; also&nbsp; did&nbsp; the chromo- genic&nbsp; assay&nbsp; for&nbsp; factor&nbsp; VIII&nbsp; and&nbsp; this&nbsp; was normal.&nbsp; Chromogenic&nbsp; factor&nbsp; assays&nbsp; are based&nbsp; on chromogenic&nbsp; substrates&nbsp; spe- cific&nbsp; for&nbsp; the factor&nbsp; assayed&nbsp; and&nbsp; are&nbsp; not phospholipid-dependent,&nbsp; hence&nbsp; will&nbsp; not be affected by the LA (Kazmi et al. 1998). The&nbsp; DRVVT&nbsp; and Staclot&nbsp; tests&nbsp; confirmed the presence of a LA in our patient. The DRVVT is very specific for LA and is not prolonged&nbsp; in&nbsp; the presence&nbsp; of&nbsp; a specific inhibitor to factor VIII or IX as the DRVVT directly&nbsp; activates&nbsp; factor&nbsp; X&nbsp; (Tropodi&nbsp; et al. 2005).</p>\r\n<p>\r\n	This patient was treated initially with re- combinant&nbsp; activated&nbsp; factor&nbsp; VII&nbsp; when&nbsp; she was misdiagnosed with factor IX inhibitor. This was unnecessary and inappropriate. Not&nbsp; only&nbsp; was&nbsp; it&nbsp; expensive,&nbsp; it&nbsp; could&nbsp; have been&nbsp; dangerous&nbsp; to&nbsp; the&nbsp; patient&nbsp; as&nbsp; she could&nbsp; have&nbsp; had&nbsp; a&nbsp; thrombosis.&nbsp; Patients with LA have an increased risk for throm- bosis&nbsp; and&nbsp; infusion&nbsp; of&nbsp; activated&nbsp; factor concentrates&nbsp; will&nbsp; increase&nbsp; this&nbsp; risk&nbsp; fur- ther.&nbsp;&nbsp;&nbsp; This&nbsp; case&nbsp; report&nbsp; illustrates&nbsp; the&nbsp; im- portance of correct interpretation of factor assays&nbsp; in&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; LA&nbsp; to&nbsp; avoid misdiagnosis&nbsp; and&nbsp; inappropriate&nbsp; treat- ment.&nbsp; In summary, a specific factor inhi- bitor&nbsp; can&nbsp; be&nbsp; differentiated&nbsp; from&nbsp; a&nbsp; lupus anticoagulant&nbsp; by:&nbsp; (1)&nbsp; the&nbsp; clinical&nbsp; history: patients&nbsp; with&nbsp; a&nbsp; specific&nbsp; factor&nbsp; inhibitor usually&nbsp; bleed,&nbsp; while patients&nbsp; with LA&nbsp; are usually&nbsp; asymptomatic&nbsp; or&nbsp; they&nbsp; may present&nbsp; with&nbsp; a&nbsp; thrombosis;&nbsp; they&nbsp; rarely bleed;&nbsp; (2)&nbsp; dilution&nbsp; assays:&nbsp; factor&nbsp; levels with&nbsp; a&nbsp; specific&nbsp; inhibitor&nbsp; should&nbsp; not&nbsp; rise with&nbsp; serial&nbsp; dilutions&nbsp; of&nbsp; patient&rsquo;s&nbsp; plasma; (3)&nbsp; DRVVT:&nbsp; a&nbsp; specific&nbsp; factor&nbsp; inhibitor&nbsp; to factor&nbsp; VIII&nbsp; or&nbsp; IX&nbsp; will&nbsp; not&nbsp; prolong&nbsp; the DRVVT; (4) mixing with phospholipids: a specific factor inhibitor should not be cor- rected&nbsp; with&nbsp; phospholipids&nbsp; and&nbsp; (5)&nbsp; chro- mogenic factor assay: factors assayed by the&nbsp; chromogenic&nbsp; method&nbsp; will&nbsp; remain&nbsp; low in&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; specific&nbsp; factor&nbsp; inhi- bitor (Douglas 1994).</p>\r\n<p>\r\n	<strong>CONCLUSION </strong></p>\r\n<p>\r\n	A&nbsp; lupus&nbsp; anticoagulant&nbsp; can&nbsp; affect&nbsp; the&nbsp; ap- parent&nbsp; levels&nbsp; of&nbsp; the&nbsp; APTT-based&nbsp; factor assays&nbsp; leading&nbsp; to&nbsp; a&nbsp; misdiagnosis&nbsp; of&nbsp; a specific&nbsp; factor&nbsp; inhibitor.&nbsp; It&nbsp; is&nbsp; important&nbsp; to distinguish&nbsp; LA&nbsp; interference&nbsp; from&nbsp; factor deficiency&nbsp; or&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp; specific factor&nbsp; inhibitor&nbsp; to&nbsp; avoid&nbsp; misdiagnosis&nbsp; be- cause the implications and the treatment differ between these groups of patients.&nbsp;</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENTS&nbsp; </strong></p>\r\n<p>\r\n	We would like to thank the Director Gen- eral of Health Malaysia for permission to publish&nbsp; this&nbsp; paper&nbsp; and&nbsp; to&nbsp; the&nbsp; director&nbsp; of Ampang Hospital, Datin Dr. Aishah Maki- nuddin&nbsp; and&nbsp; all&nbsp; staff&nbsp; of&nbsp; the&nbsp; Hematology Department for supporting this work. Our appreciation&nbsp; is&nbsp; extended&nbsp; to&nbsp; Dr&nbsp; Mike Laffan,&nbsp; Professor&nbsp; of&nbsp; Haemostasis&nbsp; and Thrombosis, Imperial College London for his invaluable input and advice on writing this manuscript.</p>\r\n', 1),
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(956, 951, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>INTRODUCTION</strong></p>\r\n<p>\r\n	Ascariasis is a common helminthic infestation in Malaysia, particularly in rural settings. Ascariasis lumbricoides normally lives in the upper small bowel without causing symptoms. Complications arise when these worms migrate into the bile duct (biliary ascariasis) causing biliary colic, cholangitis, acalculous cholecystitis and hepatic abscess. A case of biliary ascariasis presenting as biliary colic is described. Diagnosis was established using both ultrasound of the hepato-biliary system (HBS) and Endoscopic Retrograde Cholangio-Pancreatography (ERCP). Endoscopic removal of the worm led to rapid resolution of symptoms.</p>\r\n<p>\r\n	<strong>CASE STUDY: </strong>49 year old female of Indigenous origin (Orang Asli) first arrived to our district hospital for severe right upper quandrant pain for about 2 days duration. The pain was paroxysmal, radiating to the back and was accompanied by nausea and vomiting. There was no history of previous attacks of pain, jaundice or fever. On clinical examination, she was febrile (38.5) but not jaundiced. The right hypochondrium was tender with mild rigidity. Murphy&rsquo;s sign was positive. Her Blood investigation show her HB is 10.5 g/dl and Her White cell count was 13.5 with predominant 90% polymorphs, 4% eosinophils and 6% lymphocytes. Serum bilirubin: 36 umol/L. &nbsp;Her liver enzymes were within normal limits.. With the provisional diagnosis of acute cholecystitis the patient was manage conservatively with intravenous fluids, iv cefuroxime 1500mg x 8 hourly and given iv analgesia (tramadol 50 mg 6 hourly). The pain subsided for few hours but recurred with attacks of increasing severity the next day and the patient became mildly jaundiced. Liver function test were remarkable, with raised alkaline phosphatase 300 U/L. In view of these new developments, patient was referred to the nearest tertiary hospital for further investigation and management. Upon transfer over to the next setting, imaging modalities including USG HBS and CT-Abdomen were proceeded. Her USG revealed a dilated proximal CBD and right intra-hepatic ducts with tubular hyper-echoic structure and mild splenomegaly (picture-1). Her CT-Abdomen revealed distal CBD obstruction with linear density, an image compatible with worm (picture-2). In view of the above findings, ERCP was proceeded and resulted in the identifying and removal of the worm (picture-3, 4). Following the procedure, patient&rsquo;s symptoms subsided and she was subsequently discharge home well with course of oral antibiotics (Metronidazole 400mg bd) and stat dose of albendazole (400mg). She remained well on her follow-up 2 weeks later at the district hospital during the review clinic session.</p>\r\n<p style="margin-left:252.0pt;">\r\n	<strong>Discussion</strong></p>\r\n<p>\r\n	Biliary ascariasis can produce a variety of manifestations. It can present with mild or severe recurrent right upper quandrant pain and when complicated by secondary infection as ascending cholangitis(Ibrarullah et al. 2011). Recurrent attacks of cholangitis can mimic cholelithasis. Ascariasis is implicated in cholangio-hepatitis, characterized by cholelithasis (often without chole-cystolithasis), biliary duct strictures and hepatic abscess(Misra &amp; Dwivedi, 2000). The common duct stones are usually multiple, soft and black. The worm in the CBD may be alive or dead. The degenerated worm or live worm may form a nidus for biliary sand and stones. Super-added bacterial infection also leads to precipitation of bilirubin by enzymatic degradation(Misra &amp; Dwivedi, 2000). The worm may migrate into the liver where it excites granuloma formation. Degeneration of the worm in the liver with secondary infection leads to hepatic abscess. In some tropical countries, ascaris liver abscess is commoner than amoebic abscess in children, ascariasis in biliary tree can cause hemobilia and rarely acute pancreatitis due to obstruction of ampulla of vater(Adaletl et al. 2005).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Usually one or two worms are present in the biliary channels but sometime massive invasion can occur. Complications like septicemia and hepatic abscess are high in massive invasions and require early diagnosis and treatment. The commonest presentation of biliary ascanais is recurrent biliary colic (95%) which was clinically difficult to distinguish from cholelithiasis(Ortega et al. 2010). Ultrasound is a useful sensitive and specific tool for diagnosing biliary ascariasis with an accuracy of about 86% provided the radiologist is aware and alerted to the sonographic appearances. The characteristic sonographic features include: (a) a single or multiple long, linear or curved echogenic structure without acoustic shadowing; (b) a thick, long, linear or curved, non-shadowing echogenic strip containing a central, longitudinal anechoic tube, probably representing the digestive tract of the worm(Ibrarullah et al. 2011).&nbsp; ERCP is an excellent diagnostic and therapeutic tool for biliary ascariasis. At ERCP the roundworms in the biliary tree appear as smooth, long, linear or curved filling defects with tapering ends. Surgery had been the traditional method of removal of worm from the bile duct until the advent of ERCP. Endoscopic removal of worm from the bile duct is safe, minimally invasive and effective(Oswal &amp; Agarwal, 2007).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Most cases will respond to conservative measures, with the worms returning spontaneously to the intestine. This is enhanced if mebendazole is given orally to deworm the intestine. Mebendazole is preferred as it is poorly absorbed from the intestine the intestinal worms are killed but the biliary worms are spared and return to the intestine(Ortega et al. 2010). Surgery is reserved for patients who fail to respond to an adequate trial of conservative therapy or show complications like hepatic granuloma and abscess. Other indications for operation are persistent right upper quandrant pain, severe or increasing jaundice and signs of right upper quandrant peritonitis. Cases treated conservatively should be followed with intravenous cholangiogram. If persistent filling defects are demonstrable surgery or endoscopic removal is indicated(Sharma et al. 2005). &nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>References</strong></p>\r\n<p>\r\n	Adaletl, I., Selcuk, D., Guflien, M., Savafi, C. &amp; Korman, U. 2005. MRCP Findings of biliary ascariasis before and after medical treatment. Turk J Gastroenterol <strong>16</strong>(2): 98-101</p>\r\n<p>\r\n	Ibrarullah, M.D., Mishra, T., Dash, A.P. &amp; Upadhaya, U.N. 2011. Biliary ascariasis &ndash; role of endoscopic intervention. Trop Gastroenterol <strong>32</strong>(3): 210-213</p>\r\n<p>\r\n	Misra, S.P. &amp; Dwivedi, M. 2000. Clinical features and management of biliary ascariasis in a non-endemic area. <em>Postgrad Med J</em> <strong>76</strong>(891): 29-32.</p>\r\n<p>\r\n	Ortega, C.D., Ogawa, N.Y., Rocha, M.S., Blasbalg, R., Motoyama-Caiadao, A.H., Warmbrand, G. &amp; Cerri, G.G. 2010. Helminthic Diseases in Abdomen: An Epidemiologic &amp; Radiologic Overview. Radiographics <strong>30</strong>(1): 253-267</p>\r\n<p>\r\n	Oswal K, Agarwal A, 2007,&rdquo;Images: Hepato-biliary ascariasis&rdquo;, Indian Journal of Radiology Imaging, 17(2):77-78</p>\r\n<p>\r\n	Sharma, U.K., Rauniyar, R.K. &amp; Bhatta, N. 2005. Roundworm infestation presenting as acute abdomen in four cases-sonographic diagnosis. Kathmandu Univ Med J(KUMJ) <strong>3</strong>(1-9): 87-90</p>\r\n', 1),
(912, 907, NULL, NULL),
(920, 915, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">INTRODUCTION</span></p>\r\n<div>\r\n	Trauma is the global leading cause of death in the first four decades of life (ACS 2008). Uncontrolled bleeding was the major cause of trauma-related morbidity and mortality, mainly from skeletal, intra-abdominal or intra-pelvic injuries due to blunt trauma (Trikha 2011). It has been reported that up to 20% of multiple injured patients sustained pelvic injury (McCormack 2010).</div>\r\n<div>\r\n	Severe pelvic injuries even though not common remains one of the most difficult clinical problems in terms of its management (Flint 2010). The mortality rates from all types of pelvic-related injuries have been reported between six to 17% (Pohlemann 1994). It was reported that pelvic fractures accounts for approximately three percent of all skeletal injuries (Brenneman 1997; Ertel 2001; Rommens 2002; Tucker 2000). In Malaysia, of more than 300,000 reported accident cases yearly, 1.55% was associated with pelvic fracture (Sabariah et al. 2009). Injuries to the pelvic girdle and limbs made up 17.7% of major trauma and were the second commonest body region involved after head and neck (Sabariah et al. 2009).</div>\r\n<div>\r\n	High energy forces are required to injure the pelvic ring (Trikha 2011 &amp; Starr 2003). As a result, patients with high energy pelvic fractures often have associated injuries and may be hemodynamically unstable. In the case of vertical shear injury to the pelvis, patients presenting with hypotension were significantly more likely to die than those who were normotensive (44% versus 17%) (Dickson 2000). In the Burgess and Young classification, ineffectiveness of linen sheets where it depends heavily on its application, and the technique varies with different devices. Once applied, it is difficult to examine the perineum. There are invasive and non-invasive clamps. Invasive clamps need expert skills for its applications like surgeons, who most of the time are not available in the field. The problems with non-invasive clamp is it may slip during patient transportation and produces high amount of pressure to the skin which could give rise to a pressure sore over the area. As for most pelvic binders, they do not provide immobilization for the associated lower limb fractures, therefore extra splints are required, which lead to difficulty during transport. On top of that some of the binders were meant for single use, which contribute to cost-benefit issues especially for developing or under developed countries.</div>\r\n<div>\r\n	In order to address all the above issues and limitations, a team of researchers and innovators from Universiti Kebangsaan Malaysia (UKM) embarked on a project to develop a device that can answer most of the issues mentioned earlier.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Development of the new pelvic and lower limb immobilization device was initiated in 2006 by a group of researchers and healthcare providers from Universiti Kebangsaan Malaysia Medical Centre (UKMMC). The team comprises of emergency physicians, orthopedic surgeons, a senior assistant medical officer (paramedic) and an engineer. The team took seven years</div>\r\n<div>\r\n	anterior-posterior compression type (APC) injuries were associated with large amounts of blood loss. Among all trauma patients, hemorrhage is the major reversible contribution to mortality in about 42% of all cases. (ACS 2008; Heetveld 2004)</div>\r\n<div>\r\n	The source of bleeding in unstable pelvic fracture can come from the bone, vessels (venous and arterial) as well as intra-pelvic visceral injury (Flint 2010). Majority of bleeding were from small and medium size venous tributaries around the cancellous bone (Huittinen 1973). However, bleeding from the artery is the most significant contributor to massive blood loss, leading to shock and mortality (Flint 2010). Usually, when someone succumbed to pelvic injury, it was a result of multiple combined sources.</div>\r\n<div>\r\n	The goal of initial treatment for controlling hemorrhage should focus on several important aspects namely: 1) reduction of pelvic volume that lead to tamponade-like effect; 2) plugging of the major vessels through angioembolization and 3) stabilization of pelvic bone with external fixation or plating (Flint 2010). The earlier these goals are achieved the better the outcome will be. The presence of an instrument or gadget that can facilitate the achievement of these goals right from pre-hospital phase will certainly help in the management of the patient.</div>\r\n<div>\r\n	At present, pre-hospital providers use circumferential linen sheet, commercially available binders or clamps to achieve these targets. However, there are some limitations with the current instruments used. Some of the limitations of using linen are the from 2006 until 2011 to finally produce the prototype. All the processes were completed locally with collaboration between UKM and the local industry.</div>\r\n<div>\r\n	The project was started by identifying limitations and drawbacks with current available devices faced by the pre-hospital care and Emergency Department personnel. This resulted in the development of a new device that took into consideration all the following characteristic features: 1) User friendly, simple, easy to handle, and safe to use; 2) Reproducible (in large quantities); 3) Light; easy to carry and store; 4) Cheap; 5) Reusable; 6) Radiolucent; 7) Tough; 8) One device fits all; 9) Value added; 10) Allow access for life-saving surgical procedures; and 11) Availability of pressure measuring and monitoring.</div>\r\n<div>\r\n	For 1) User friendly, simple, easy to handle, and safe to use: it should be an uncomplicated device which does not require a special qualification for its use that would not cause harm. 2) Reproducible (in large quantities): the device is reproducible, can be easily manufactured and does not need high technology in order to keep the cost down. 3) Light, easy to carry and stored: the devices have to be light and stored without taking up much space, especially in the ambulance and is suitable for pre-hospital setting. 4) Cheap: the material used to develop this product should be locally available and cheap yet durable. 5) Reusable: the product should be reusable which can reduce the cost. 6) Radiolucent: This device should not obscure with radiological imaging studies. 7) Tough: The material itself should be strong to withstand the force applied. 8) One device fits all: a single device that caters for both pelvic as well as lower limb fractures. 9) Value- added: This device also addresses immobilization for a concomitant lower limb fracture. 10) Allow access for life-saving surgical procedures: it should allow life-saving hemorrhage control and structure stabilization like angio-embolization and external-fixation of the pelvis. It also should allow adequate access for emergency laparotomy to be performed. 11) Availability of pressure measuring and monitoring: compression against pressure points needs to be monitored, this help to reduce the chance of pressure necrosis.</div>\r\n<div>\r\n	The development of the prototype was divided into two phases. In phase one, we concentrated on the design of the device to achieve the characteristic features listed above. In phase two, the emphasis was on obtaining the appropriate material and assessing its biomechanical properties to fit its function.</div>\r\n<div>\r\n	Phase One: Design</div>\r\n<div>\r\n	Based on the discussion from the team members, the basic structure of the device follows a &lsquo;double-I&rsquo; (Figure 1A) construct for the main frame. This frame consists of the &lsquo;base&rsquo; for the pelvic area, and two &lsquo;arms&rsquo; for the thighs. The arm piece is connected to the base, making the whole design into one piece. For the base component, a double tightening belt was attached to encircle the device. Each of the arms, which are meant to hold the thigh, has a single tightening belt, and three longitudinal sleeves were made on each arm for placement of a thermoplastic piece, which acts as a strut in order to provide longitudinal strength. The technical drawing was then given to the private manufacturer for production. The device was made of Poly Vinyl Chloride (PVC) and weighs less than one kilogram.</div>\r\n<div>\r\n	There were a few limitations noted with this first prototype, namely: the design; material used; size; locking mechanism; accessibility for life-saving procedures, lack of pressure measuring-monitoring and leg immobilization component.</div>\r\n<div>\r\n	Phase Two: Scientific testing</div>\r\n<div>\r\n	Based on the limitations encountered at the end of phase one, the research team developed a new improved design of the prototype (Figure 1B) through several studies, which includes anthropometric study, biomechanical study and radiolucency test. Biomechanical study comprised of device tensile strength, buckle pull-out strength, and a pressure distribution study. As a result of all these studies, a new prototype was produced.</div>\r\n<div>\r\n	An anthropometric study was performed on 30 subjects of different body habitus measuring several points around the pelvis. The measurements taken include hip circumference, mid-line to Anterior Superior Iliac Spine (ASIS); mid-line to femoral artery; ASIS-to-ASIS; and greater trochanter-to-greater trochanter. The results of this study will be included in another publication.</div>\r\n<div>\r\n	The pressure-distribution study was performed among three healthy subjects in the Department of Electrical, Electronic and Systems Engineering, Faculty of Engineering and Built Environment, UKM. Several regions around the pelvis were assessed, this includes bony prominents in the right and left flank of the pelvis and over the sacral area as shown in Figure 2. The data were acquired from six regions for 50 seconds and the sampling rate is one Hertz. The average reading from each region was tabulated.</div>\r\n<div>\r\n	For the biomechanical properties of the device, the test was done in the Department of Mechanical &amp; Materials Engineering, Faculty of Engineering and Built Environment, UKM. Tensile strength test was performed on the &lsquo;base&rsquo; section of the device main frame. Ten readings were observed. The buckle pull-out test was performed on the device locked-buckle that was made of high density plastic using a similar technique. Five readings were observed.</div>\r\n<div>\r\n	The device was also exposed to several radiological exposures to assess its radiolucent properties.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	At the end of Phase 2, the team managed to produce a new pelvic and lower limb immobilizer which was a two-in-one immobilization device. It not only enable immobalization of the fractures but also facilitated life-saving procedures to be carried out while maintaining the stability of the fractures.</div>\r\n<div>\r\n	The final product was a &lsquo;double-J&rsquo; construct (Figure 1B) with three different sizes that caters sizing issues. The final prototype of the new device (Figure 3) has a base and two arms that form the main frame. It has two main straps, which was the Greater Trochanter (GT) strap and the Iliac (I) strap, which is meant to secure the new device to the body. The GT strap is the main strap, placed at the level of the patient&rsquo;s greater trochanter, to produce the compressive effect of the new device. It has two apertures located at the flank of the anterior part of the device&rsquo;s main frame for insertion of the external fixator pin. It&rsquo;s anterior portion can be folded down anteriorly to give more space for a surgeon to performe laparotomy while it still provides immobilization of the patient. As a result of the &lsquo;J&rsquo; shape limb instead of &lsquo;I&rsquo;, femoral vessel catheterization for angioembolization can be performed while maintaining compressive forces.</div>\r\n<div>\r\n	There were straps to secure the device&rsquo;s arm to the patient&rsquo;s leg, and this is fortified with a thermoplastic strut in order to provide a splinting effect for the femur. Limb immobilization extenders (LIE) are attached to the main frame arm via Velcro in order to give further immobilization distal to the knee until the foot.</div>\r\n<div>\r\n	It weighs less than one kilogram and is made of polyurethane (PU) material. The strap was made of nylon while the lock-buckle is made of high density plastic. All of these features were to ensure that the whole device was radiolucent.</div>\r\n<div>\r\n	The results of the pressure distribution study on the left, middle and right pelvis at six different locations are presented in Table 1. Figure 4, 5 and 6 showed the pressure distribution for 50 seconds at six different locations on the left, middle and right pelvis.</div>\r\n<div>\r\n	Results of the biomechanical properties of the device which include tensile strength and buckle pull-out strength of the device was shown in Table 2.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The development and production of the prototype for the pelvic and lower limb immobalization device took a long time because of several factors that were encountered. One of the main limiting factors was due to financial constraint where the first prototype was built without funds obtained from any sources. It was only in the second phase that some financial support was obtained from the university research fund.</div>\r\n<div>\r\n	Some of the factors that were improved from the first phase study was the design; material used; size; locking mechanism; accessibility for life-saving procedures, lack of pressure measuring-monitoring and leg immobilization component.</div>\r\n<div>\r\n	There was a problem with accessibility for surgeon or interventional radiologist to perform life-saving procedures to help secure haemostasis. As a result of limited accessibility, the design was changed from a &lsquo;double-I&rsquo; to a &lsquo;double-J&rsquo; construct. This new design allowed procedures like external fixation and femoral catheterization for angioembolization to be performed. In addition, two openings were made on the anterior part of the base component which created a flap and it can be flipped down thus offering more accessibility for abdominal procedures like laparotomy especially for posterior pelvic packing in a pelvic fracture patient (Flint 2010).</div>\r\n<div>\r\n	Financial reasons also contributed to the decision to used PVC in the first prototype. PVC, a non-breathable material was found to be uncomfortable to the patient since it trapped heat. A suitable substitute flexible polymer, polyurethane (PU), was used to replace the PVC, which would address the fore mentioned issues. Strength, durability and skin-friendliness were achieved with the new material after the biomechanical testing and discussion with material experts.</div>\r\n<div>\r\n	Other problems that we encounter at the end of the first phase were the sizing issue. One size will not fit the whole population. For this reason an anthropometric study was performed in the second phase to determine the best size that could fit all or most of the population. One-size-fits all was the &lsquo;ideal characteristic&rsquo; wish-list, but from our study we found that we need at least three sizes to fit the population at large.</div>\r\n<div>\r\n	The initial locking mechanism was not user-friendly since the buckle tends to give way with minimal force. The buckle was changed to allow a better locking mechanism that minimizes toggling and buckle pullout. The new buckle helps to improve the buckle pull-out strength, where in the biomechanical study an average load of more than 750 Newton (N) was needed to create a pull-out. This was sufficient since most of the time a tensile force lesser than that is sufficient to bind the pelvis.</div>\r\n<div>\r\n	Though the first prototype was able to immobilize the pelvis and the thighs, it did not immobilize a fracture of the lower legs (tibia&ndash;fibula). Therefore, a lower limb immobilization extension (LIE) was created with additional traction capability. These features help stabilization of the lower limbs better and will enable easy transportation and care especially in the prehospital setting.</div>\r\n<div>\r\n	The amount of pressure applied to the skin was not measurable in the first prototype since it does not incorporate a pressure measurement device. From our pressure distribution study, we also noted that the area at the posterior aspect of sacrum was very high (72.9-79.9 kPa). A sleeve was created at the posterior base component to enable an air cushion-pad to be slipped in and off-load the higher pressure at this area thus preventing pressure sores on prolonged use. We planned in our next prototype to incorporate a pressure gauge meter connected to the air-cushion pad that can measure pressure continuously and at the same time off-load the pressure area.</div>\r\n<div>\r\n	Another important aspect in the creation of the new device pertaining to the material used was to ensure that the whole device was radiolucent since it was very essential for use during patient management. That was the reason why only radiolucent-friendly material was used throughout the whole device.</div>\r\n<div>\r\n	Radiological testing proves its radiolucent property (Figure 7). The material used allows the device to be washed and cleaned with decontamination solution, making it reusable. The new device was assembled locally with minimal cost.</div>\r\n<div>\r\n	Following a series of modification and improvement of the initial design, the first prototype was patented in 2008 (IP Pattern: PI 2008 1332&ndash;10th JUNE 2008) while the second was filed for patency in 2011 (IP Patent: PI2011004592-27th September 2011).</div>\r\n<div>\r\n	SUMMARY</div>\r\n<div>\r\n	A new pelvic and lower limb immobilization device, BRIMTM Immobilizer, was designed and produced according to the characteristic wish-list that incorporated suitable but relatively affordable material with a design that allows life saving intervention to be carried out while the patient is still on it. It addresses some of the local problems faced by our prehospital care providers with the current treatment methods. It is also radiolucent, reusable, light but yet tough.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The second phase of the study was fully funded by a research grant from the Universiti Kebangsaan Malaysia (research project code: UKM-IF-1-10-005 UKM innovation fund 2010). We would like to extend our appreciation to the staffs of the Department of Emergency Medicine UKMMC involved with the projects directly or indirectly. We would also like to thank Ms Nur Solehah Mohtar, Ms Norhidayu Mohd Norapi and Ms Nur Hazwani Mamat for helping in the study. Special thanks to Prof. Dr-Ing. Nik Abdullah Nik Mohamed, Department of Mechanical &amp; Materials Engineering, Faculty of Engineering and Built Environment, Prof. Dr. Ahmad Kamal Ariffin Mohd. Ihsan, Department of Mechanical &amp; Materials Engineering, Faculty of Engineering and Built Environment and Prof. Dr. Mohd. Alauddin bin Mohd. Ali, Instute of Space Science (ANGKASA) for their undivided support.</div>\r\n', 1),
(905, 900, NULL, NULL),
(908, 903, NULL, NULL),
(933, 928, '<p>\r\n	&nbsp;</p>\r\n<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Graves disease (GD) is a thyroid-specific autoimmune disease characterised by the presence of autoantibodies which binds to the TSH receptor (TRAb) and leads to overactivity of the thyroid gland which clinically manifest as hyperthyroidism. Besides that, the inflammation of the eye muscles by attacking autoantibodies give rise to Graves orbitopathy (GO).</div>\r\n<div>\r\n	The detection of TRAb is valuable in confirming the diagnosis of GD in mild hyperthyrodism, euthyroid Graves ophtalmopathy without goiter, differentiating GD from other toxic nodular goiter or facticious thyrotoxicosis (Weetman et al. 2000) and recent studies have shown that TRAb detection is potentially useful in predicting outcome in patient with GD and GO respectively. (Smith et al. 2007) The measurement of TRAb has been shown to be valuable in determining the causes of post partum thyrotoxicosis either due to post-partum thyroiditis or Graves disease. Furthermore this marker is useful in predicting the likelihood of either foetal or neonatal thyrotoxicosis in women undergoing<span style="font-size: 1.2em; line-height: 1.7em;">treatment or those who had thyroid ablation (Saravanan &amp; Dayan 2001).</span></div>\r\n<div>\r\n	In tandem with advance in technology, great improvement has been achieved in TRAb detection methodologies. At present, the bioassay and competitive TSH inhibition binding assays have been established. (Sanders et al. 2002; Scott et al. 2005; Smith et al. 2007). Among them, the competitive assays are the only validated routine test for TRAb analysis. These assays are based on the ability of TRAb to inhibit TSH receptor (TSHR) binding of labelled bovine TSH because purified bovine TSH has higher affinity towards TSH receptor than human TSH and it is a more suitable ligand for 125I or biotin labelling (Morgenthaler 1999). An alternative to bovine TSH have been available, for example, labelled mouse thyroid-stimulating monoclonal antibodies with similar performance (Sanders et al. 2002). More recently, Smith et al. (2005) developed an enzyme-linked immunosorbent assay (ELISA) using a labelled human thyroid stimulating monoclonal autoantibody M22 (Smith et al. 2005). In this assay, TRAb are detected based on their ability to inhibit the binding of labelled human monoclonal thyroid stimulating antibody (M22) to porcine TSH-coated ELISA plates.</div>\r\n<div>\r\n	Three comparison studies have been published (Kamijo 2003; Kamijo 2007; Liu et al. 2008) and two studies have found that the M22-biotin based ELISA have excellent sensitivity and specificity (Kamijo 2003; Kamijo 2007) while the remaining study showed relatively poor precision (CV&gt;20) (Liu et al. 2008).</div>\r\n<div>\r\n	As with other manual assays, the drawback of this ELISA assay is on their sensitivity, specificity and reproducibility. Furthermore, the typically long incubation times and intense manpower needed to perform this assay, make it unpopular in busy service laboratories.</div>\r\n<div>\r\n	Recently, a rapid (27 minutes), fully automated electrochemiluminescent immunoassay ElecsysAnti-TSHR for detection of autoantibodies to TSH receptor was made available for routine clinical use. The methodology of this assay incorporated the used of solubalized porcine TSH receptor immunocomplexed with a biotynylated mouse monoclonal antibody to the porcine TSH receptor and M22 human monoclonal antibody as a ruthenium- labelled assay ligand.</div>\r\n<div>\r\n	The aim of this study was to evaluate the ElecsysAnti-TSHR assay system in terms of reproducibility, sensitivity, specificity and to determine the cut-off value of this assay and the feasibility of this assay to be offered as a routine laboratory test for UKM Medical Centre (UKMMC).</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	The study was conducted in the Chemical Pathology Laboratory, Department of Diagnostic Laboratory Services, UKMMC.</div>\r\n<div>\r\n	Sample collection</div>\r\n<div>\r\n	The samples were acquired from the Endocrinology and Chemical Pathology laboratories and the diagnosis of the patients were based on the patients&rsquo; record from the hospital or laboratory information system.</div>\r\n<div>\r\n	Inclusion criteria: Samples from patients with thyroid disorders which include GD, Hashimoto thyroiditis (HD), non autoimmune nodular goitre (NAG), thyroid cancer (Ca).</div>\r\n<div>\r\n	Reproducibility study (Coefficient variation, CV):</div>\r\n<div>\r\n	The intra-assay imprecision CV was determined by 21 replicates in a single run on Cobas e411 electrochemiluminescent immunoassay analyzer using a manufacturer control material of two levels PreciControl Thyro 1 (PCThyro 1 range: 3.78-7.02 IU/L ) and and PreciControl Thyro 2 (PCThyro 2 range: 13.5-21.2 IU/L).</div>\r\n<div>\r\n	Total imprecision CV study was performed according to CLSI/NCCLS guideline EP5 A2 using aliquots of control materials PC Thyro 1 (range: 3.78-7.02 IU/L) and PC Thyro 2 (range: 13.5-21.2 IU/L) which were analysed in two determinations per run and two runs per day for five days.</div>\r\n<div>\r\n	Sensitivity, specificity and cut-off value:</div>\r\n<div>\r\n	The determination of optimal decision threshold level for positivity by receiver-operating characteristic (ROC) analysis was performed using serum from apparently healthy subjects, patients with GD and patients with other thyroid diseases which were acquired retrospectively.</div>\r\n<div>\r\n	Duration of assay performance</div>\r\n<div>\r\n	To validate the claim by the manufacturer that the total duration of this assay is 27 minutes, we examined the time from the samples was loaded in the analyzer and the time the result was made available by the analyzer.</div>\r\n<div>\r\n	Test principle</div>\r\n<div>\r\n	The ElecsysAnti-TSHR assay is a third generation assay using a preformed immunocomplex based on native porcine TSHR solubalised from a thyroid cell membrane preparation and biotinylated anti-porcine TSHR mouse monoclonal capture antibody and a ruthenium labeled human TSHR stimulating monoclonal detection antibody (M22). The capture antibody binds to the C-terminal moiety of the porcine TSHR (Bolton et al. 1999) which does not interfere with the binding of TRAb or M22 to the TSHR. After addition of streptavidin-coated microparticles and ruthenium labeled M22, serum TRAb bound to the TSH receptor are detected by the ability to inhibit the binding of labeled M22. The entire complex becomes bound to the solid phase via interaction of biotin and streptovidin. Anti-TSHR is calibrated against the National Institute for Biological Standards and Control (NIBSC) 90/672 standard. The test measuring range was 0.3-40 IU/l. The within run CV for PCThyro 1 and PCThyro 2 were 3.1 and 1.4 respectively and the total CV for PCThyro 1 and PCThyro 2 were reported at 5.5 and 2.4 respectively. The cut-off value was 1.75 IU/l. However, each laboratory should investigate the transferability of the expected values to its own patient population and if necessary determine its own reference range.</div>\r\n<div>\r\n	Statistical analysis</div>\r\n<div>\r\n	All clinical and laboratory data were stored and analyzed using the Statistical Package for Social Science (SPSS) software version 17.0.</div>\r\n<div>\r\n	A receiver operator characteristic (ROC) curve was constructed to determine the sensitivity, specificity and cut-off value.</div>\r\n<div>\r\n	In all statistical analyses, p &lt; 0.05 (95% confidence interval) were considered significant.</div>\r\n<div>\r\n	RESULTSReproducibility study (Coefficient variation CV):</div>\r\n<div>\r\n	The mean value for PreciControl Thyro 1 and 2 were 3.90 IU/l (range: 3.78-7.02 IU/l and 20.80 IU/l (range 13.50-21.20 IU/L) respectively. The intra-assay CVs were 2.4% and 0.8% for level PreciControl Thyro 1 and 2 respectively. The total imprecision were 3.8% and 1.0% for PreciControl Thyro 1 (mean: 3.8 IU/l, range: 3.78-7.02 IU/l) and PreciControl Thyro 2 (mean: 20.80 IU/l, range 13.50-21.20 IU/L). Results of the reproducibility study are summarized in Table 1.</div>\r\n<div>\r\n	Sensitivity, specificity and cut-off value:</div>\r\n<div>\r\n	A total of 124 samples which comprised of 46 GD (29 newly diagnosed GD and not on antithyroid therapy), 7 HD, 11 NAG, 2 Ca and 58 normal controls were retrospectively analysed to determine the sensitivity, specificity and cut-off value. The patients&rsquo; general characteristics are shown in Table 2. The samples were divided into two cohorts; sensitivity cohort comprises of the GD patients and the specificity cohort are made up by 20 patients with other thyroid diseases and 58 normal control subjects. The mean age of the study population was 40.3&plusmn;16.1 years (range: 14-69 years). There was significant difference between the age of the patients in the sensitivity cohort and those in the specificity cohort, (35.3&plusmn;15.7 years vs 42.7&plusmn;16.4 years, (p&lt;0.05)). Female preponderance was seen in both sensitivity and specificity cohorts. The majority of the study population was Malay followed by Chinese and Indian in both cohorts. (Table 2)</div>\r\n<div>\r\n	The ROC analysis showed an area under curve (AUC) of 0.99 (95% Cl: 0.98-1.0). The optimal pair of sensitivity (94%) and specificity (98%) was found at a TRAb level of 1.69 IU/l (Figure 2). Positive predictive value (PPV) and negative predictive value (NPV) was 95% and 94% respectively</div>\r\n<div>\r\n	Duration of assay performance</div>\r\n<div>\r\n	The mean total duration of the assay was 25.70&plusmn;0.02 minutes (n=124). This finding validated the manufacturer&rsquo;s claim of the total assay duration of 27 minutes.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The diagnosis of Graves hyperthyroidism is based on the clinical presentation and biochemical changes of hyperthyroidism. Traditionally, serum TSH and free T4 are measured to confirm the diagnosis of hyperthyroidism.</div>\r\n<div>\r\n	The measurement of serum TRAb is not routinely done because the diagnosis of GD can nearly always be made correctly based on the clinical findings. Furthermore, the inability of the routine laboratory to perform this test partly due to the facts that different methodology gives different sensitivity and specificity and the test method can be cumbersome. With great improvement in TRAb detection methodologies, this test can be done routinely in most laboratories.</div>\r\n<div>\r\n	We evaluated the third generation, fully automated electrochemiluminescent immunoassay Elecsys&acirc;Anti-TSHR for detection of autoantibodies to TSH receptor.</div>\r\n<div>\r\n	From this study, the intra-assays and total imprecision CVs were found to be less than 5%. This finding showed that Elecsys&acirc;Anti-TSHR assay meets the requirement CVs of less than 20%. (Hermsen et al. 2009)</div>\r\n<div>\r\n	With regards to the cut-off value, the optimal pair of sensitivity (94%) and specificity (98%) was found at TRAb level of 1.69 IU/L. The calculated Positive predictive value (PPV) and Negative predictive value (NPV) were 96% and 97% respectively. We further studied the 29 newly diagnosed GD patients; at the derived cut-off value of 1.69IU/L, we found that the sensitivity of TRAb assay was 94%.</div>\r\n<div>\r\n	Our finding was different from the manufacturer expected value and other studies (Hermsen et al. 2009; Schott et al. 2009) which reported the cut-off value as 1.75 IU/l. When we used the calculated cut-off for TRAb positivity of 1.75 IU/l, we found the sensitivity and specificity of 87% (95% CI: 0.86- 0.93) and 99% (95% CI: 0.97- 0.99).</div>\r\n<div>\r\n	This discrepancy in the cut-off value could be due to the difference in the studied population and small sample numbers. In this study we included the GD which already received antithyroid drug therapy including GD patient on long-term follow up, while in the study by Hermsen et al. (2009) only the untreated GD patients were included as sensitivity cohort. We included this group of patient to simulate the requirements in clinical routine when patients are included with long-lasting history of GD and potential negative TRAbs. Schott et al. (2009), performed two sets of ROC analysis, the first analysis include only the untreated GD patients as sensitivity cohort whilst the other include all the GD patients; they found that at 1.75IU/l, lower sensitivity and good specificity were found when all GD patients were analysed together; a finding which was similar to ours.</div>\r\n<div>\r\n	From this study, we concluded that the fully automated electrochemiluminescent immunoassay ElecsysAnti-TSHR assay with testing time of 27 minutes has excellent reproducibility with high sensitivity in detecting GD and high specificity for discriminating other thyroid diseases. Moreover, with the rapid turn-around time and less man power needed than the manual assay, it is feasible to be offered as a routine laboratory test and represent a major improvement in the diagnosis and management of patients with thyroid diseases.</div>\r\n<div>\r\n	Authors&rsquo; disclosures of Potential Conflicts of Interest: All authors completed and signed the disclosure conflict of interest form before conducting this study.</div>\r\n<div>\r\n	Role of sponsor: The funding organization play no role in the design of this study, choice of enrolled patients, review and interpretation of data or preparation or approval of this manuscript.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT.</div>\r\n<div>\r\n	This study was supported by Roche Diagnostics.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(936, 931, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">INTRODUCTION&nbsp;</span></p>\r\n<div>\r\n	In Malaysia, breast cancer is the commonest cancer in all ethnic groups, accounting for 30.4% of newly diagnosed cancer cases in Malaysian women (Lim et al. 2003). The incidence of breast carcinoma increases from year to year and the risk rises with increasing age.</div>\r\n<div>\r\n	Until now, the true aetiology of breast carcinoma is still unknown. However, studies have shown that it is a multifactorial and multigenic disease. Genetic factors are one of the risk factors that have been identified or believed to cause breast carcinoma. Several studies have been done to identify the role of biologic tumour markers in predicting response towards treatment of breast carcinoma. Of all the biologic tumour markers that have been identified, the relationship between p53 and bcl-2 protein expression has become the focus of attention.</div>\r\n<div>\r\n	Breast carcinogenesis involves at least one initiation stage and promotion or progression stage. It can be associated with epigenetic alterations and genetic response which controls gene regulation. Among these alterations, deactivation of p53 gene is considered the most common genetic change (Schmitt et al. 1995).</div>\r\n<div>\r\n	The p53 gene is a tumour suppressor gene which is located on the short arm of chromosome 17 at 17p13.1. It encodes the 53kDa nuclear phosphoprotein expressed by normal cells and contains many important biological functions. This includes growth and cellular transformation, cell cycle and genomic stability. It also regulates apoptosis which is important after DNA damage.&nbsp;</div>\r\n<div>\r\n	Abnormalities identified in the p53 tumour suppressor gene are some of the molecular incidences seen in human and animal neoplasms (Hainaut et al. 1998). P53 is involved in regulation of cell proliferation, directing apoptosis and promoting chromosomal stability. Any disturbances in these functions play an important role in carcinogenesis (Levine 1997). Changes in the p53 gene cause it to lose its functions as a cell growth regulator.</div>\r\n<div>\r\n	Somatic mutation of p53 is one of the molecular abnormalities which is frequently seen in carcinoma (Coles et al. 1992; Osborne et al. 1991). Previous studies have shown that somatic gene mutation of p53 was present in 30% to 50% of breast carcinoma. It has been proven that protein overexpression of mutant p53 gene is associated with a poor response towards chemotherapy and poor prognosis in patients with breast carcinoma (Davidoff et al. 1991; Kovach et al. 1996; Isola et al. 1992; Silvestrini et al. 1993; Thor et al. 1992).</div>\r\n<div>\r\n	Bcl-2 is a human proto-oncogene and is located on chromosome 18 at 18q21. Bcl-2 produces the membrane integral protein which is located on the endoplasmic reticulum membrane, nuclear envelope and outer mitochondrial membrane. The location of Bcl-2 suggests that it may be involved in some of the pathways of metabolic function of mitochondrial membrane (Hockenbery et al. 1990).</div>\r\n<div>\r\n	Bcl-2 was first discovered in follicular and diffuse lymphomas possessing t(14;18) chromosomal translocation and has been established as a key regulator of apoptosis (Kroemer 1997). A decreased expression of Bcl-2 protein was shown to be associated with a poor clinical outcome in breast cancer (Silvestrini et al. 1994; Joensuu et al. 1994; Gasparini et al. 1995).</div>\r\n<div>\r\n	An inverse relationship between p53 and Bcl-2 has been shown in primary breast carcinomas, and the two oncoproteins may interact in the regulation of apoptosis (Silvestrini et al. 1994; Gasparini et al. 1995; Barbareschi et al. 1996). Recently, p53 has been shown to be inactivated by alterations other than mutation of TP53 itself (Abdel-Fatah et al. 2010). More importantly, completely inactive p53 pathway exhibited unfavorable clinicopathologic features, bad prognosis and poor outcome despite receiving systemic adjuvant therapy (Abdel-Fatah et al. 2010). However, studies investigating the relationship between p53 and Bcl-2 have mainly been in breast cancers involving the western population. To date, there have been no studies performed on the significance of p53 and Bcl-2 expression in breast carcinomas in Malaysian women.</div>\r\n<div>\r\n	Based on these observations, we aim to study the expression of p53 and one of its downstream genes, Bcl-2 and its role in tumorigenesis of breast carcinomas in Malaysian patients. We also aim to correlate the findings with other prognostic variables and clinicopathological data.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Breast Cancer Specimens</div>\r\n<div>\r\n	This is a retrospective study utilising paraffin embedded tissue (PET) blocks from 59 cases of breast carcinomas (53 infiltrating ductal carcinoma, 2 ductal carcinoma in situ, 2 mucinous carcinoma, 1 squamous cell carcinoma, 1 papillary carcinoma) at Universiti Kebangsaan Malaysia Medical Centre (UKMMC). This study was approved by the Ethics Committee of UKMMC. Clinical data of the patients were obtained from the Medical Records Unit, UKMMC. Only cases with complete medical data were selected. Haematoxylin and eosin stained slides from the tumour of each of the cases were reviewed. Sequential sections for further immunohistochemical studies were determined after selection of the appropriate tumour section.&nbsp;</div>\r\n<div>\r\n	Immunohistochemical Study</div>\r\n<div>\r\n	Three &micro;m thick sequential sections from PET blocks were cut and placed on poly-L lysine coated slides. The slides were then placed in an oven for 30 minutes at 55˚C. The tissue sections were then dewaxed in xylene and rinsed in graded alcohol. Endogenous peroxidase was blocked by incubation in 3% hydrogen peroxide. Subsequently the slides were rinsed in running water before being subjected to Target Retrieval Solution for 20 minutes at 97˚C. For p53 staining, a Target Retrieval Solution of normal pH was used while a high pH was used for Bcl-2 staining. The slides were then allowed to cool for 20 minutes and washed in TBS. The primary antibody is then dropped onto the tissue sections before being incubated for 30 to 60 minutes, followed by a rinse in TBS. The slides are then incubated for 15 minutes with link antibody. Subsequently streptavidin and peroxidase were applied for 15 minutes. The immunoprecipitate was visualized by treating with Diaminobenzidine (DAB) and counterstaining with haematoxylin.</div>\r\n<div>\r\n	Controls&nbsp;</div>\r\n<div>\r\n	Known p53 and Bcl-2 positive breast carcinomas were used as positive controls for p53 and Bcl-2 respectively. Negative controls were run with each batch by replacing the primary antibody with normal saline.</div>\r\n<div>\r\n	Interpretation of Results and Staining Characteristics</div>\r\n<div>\r\n	Each case was assessed by two observers without the knowledge of patient&rsquo;s clinical data. The tumour cells showed positive nuclear staining with p53. Cases were considered positive for each marker when at least 5% of the cells showed positive staining with the relevant antibody (Poller et al. 1993).</div>\r\n<div>\r\n	Positive Bcl-2 staining was cytoplasmic and tissue sections showing less than 10% of the cells with positive staining were considered negative (Simon &amp; Altman 1994).</div>\r\n<div>\r\n	Statistical Analysis</div>\r\n<div>\r\n	The relationship between Bcl-2 and p53 immunostaining, and with other clinicopathological variables were evaluated using the X2-test. Statistical procedures were performed using the SPSS software version 10.0.1. The statistical significance was defined as p&lt;0.01.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Twenty seven of 59 (45.7%) of the cases showed p53 immunopositivity (Table 1, Figure 1). Heterogenous and homogenous distribution pattern were seen with p53 nuclear staining. Eleven out of 27 cases showed heterogenous distribution of p53 immunostaining while 16 cases showed homogenous distribution pattern. &nbsp;Both nuclear and cytoplasmic staining were seen in four cases.</div>\r\n<div>\r\n	Twenty four of 59 (40.7%) of the cases showed Bcl-2 immunopositivity (Table 2, Figure 2). The distribution pattern for Bcl-2 immunostaining was mostly homogenous. However, five out of 24 cases showed heterogeneous distribution pattern of Bcl-2 immunostaining. Bcl-2 immunostaining was also present in the normal breast epithelial cells adjacent to the tumour cells in five cases.</div>\r\n<div>\r\n	Results also showed a significant inverse relationship between p53 and Bcl-2 protein expressions in the tumours studied (X2=10.130b, df=2, p=0.001). Thirteen of 59 (22%) cases are immunonegative for both p53 and Bcl-2. Only five cases were immunopositive for both p53 and Bcl-2 proteins. Twenty two cases (37.3%) were immunopositive for p53 and immunonegative for Bcl-2. Nineteen cases (32.2%) were immunopositive for Bcl-2 and immunonegative for p53 (Table 3). Neither p53 nor Bcl-2 expression showed any significant correlation with tumour size, age and histologic grade (Table 4).&nbsp;</div>\r\n<div>\r\n	There was no association seen between p53 expression and individual disease stage (Table 5). On the contrary, a significant association was found between Bcl-2 expression and early stage of disease (Stage 0, I, II). Six of seven (85.7%) patients with early stage disease showed Bcl-2 immunopositivity (p=0.01) while only 34.6% of patients with late stage disease (stage III, IV) showed Bcl-2 immunopositivity (Table 6).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	p53 and Bcl-2 are known to be involved in the regulation of apoptosis and have been investigated widely in neoplasms (Silvestrini et al. 1994; Watson et al. 1996). The loss of p53 function represents the most common genetic change in human neoplasms. Experimental studies have demonstrated Bcl-2 to be implicated in regulating the cell cycle and proliferation (Watson et al. 1996; Bonnefoy-Berard et al. 2004). Both p53 and Bcl-2 are expressed in breast cancer and have been shown to associate with a series of clinicopathological parameters (Watson et al. 1996). However, the relationship between p53 and Bcl-2 expression and their role in breast tumorigenesis in Malaysian patients has not been reported.</div>\r\n<div>\r\n	In this present study, there is an overexpression of p53 in 45.8% of the cases. This is consistent with the findings of other previous studies for which the range was between 16% to 58% (Davidoff et al. 1991; Isola et al. 1992; Thor et al. 1992; Bartek et al. 1990; Molina et al. 1998; Allred et al. 1993). The variation of p53 expression in the different studies is probably attributed by the number of samples studied, preferred methodology and the types of antibody that was used.</div>\r\n<div>\r\n	Studies of p53 expression on ductal carcinoma in situ (DCIS) are limited (Davidoff et al. 1991; Thor et al. 1992; Poller et al. 1993; Walker et al. 1991). In our study, one of the two DCIS cases showed positive expression of p53. This finding is in keeping with previous studies and supports the idea that abnormal p53 protein is implicated in the early development of breast carcinomas (Walker et al. 1991).</div>\r\n<div>\r\n	We also found that Bcl-2 expression was present in 40.7% of the cases. This finding is in keeping with the findings of other previous studies for which the range of expression varies between 38.7% and 81.2% (Joensuu et al. 1994; Charpin et al. 1998; Berardo et al. 1998; Coppola et al. 1999). The variation of Bcl-2 expression in the different studies is probably attributed by tissue sampling (paraffin or frozen section), method of analysis, and the positive cut- off point.&nbsp;</div>\r\n<div>\r\n	In this study, Bcl-2 expression was identified in almost all of the normal breast epithelia adjacent to the tumours. This suggests that Bcl-2 expression is required to protect normal breast epithelial cells from apoptosis and ensure the survival of the breast epithelium (Zhang et al. 1997).</div>\r\n<div>\r\n	Both of the DCIS cases in our study had shown Bcl-2 expression whereas only 35.8% of the invasive breast carcinomas had shown Bcl-2 expression. A significant decrease in Bcl-2 expression was observed during the evolution from normal breast epithelial cells to ductal carcinoma in situ (DCIS) and from DCIS to invasive breast carcinomas. This present result indicates that the reduction in Bcl-2 expression is associated with carcinogenesis and progression of breast carcinoma. This phenomenon was also reported in other studies (Zhang et al. 1997; Leek et al. 1994). The down regulation of Bcl-2 is necessary to induce apoptosis of cancer cells for the maintenance of increased cell turnover and the balance between tumour growth and apoptosis. Although we could not find any significant correlation between Bcl-2 expression and DCIS due to the limited number of DCIS cases studied, our findings suggest that DCIS may share smilar molecular pathways to that of an invasive carcinoma. Of note, the lack of Bcl-2 expression was seen more in recurrent DCIS (Provenzona et al. 2003), suggesting the role of Bcl-2 in the progression and malignant transformation of DCIS.</div>\r\n<div>\r\n	The present study had demonstrated a significant inverse correlation between p53 and Bcl-2 expressions in breast carcinoma. A significant inverse correlation was also demonstrated in other studies (Kroemer 1997; Silvestrini et al. 1994; Joensuu et al. 1994; Gasparini et al. 1995; Barbareschi et al. 1996) while no correlation was found in others (Bonnefoy-Berard et al. 2004; Nohara et al. 2001; Lau et al. 2001; Sgambato et al. 2000). Haldar and colleagues had shown that the overexpression of mutant p53 in breast cancer (MCF-7) cell line induced the down regulation of Bcl-2 at both the protein and mRNA levels (Haldar et al. 1994).</div>\r\n<div>\r\n	We have also demonstrated that 37% of p53 immunopositive breast cancer were Bcl-2 immunonegative. In addition, we have also found that lack of Bcl-2 expression was also more associated with tumours of late stage. A combination of p53 overexpression and lack of Bcl-2 expression was more likely to be seen in aggressive tumours with reduced five year survival (Rolland et al. 2007).</div>\r\n<div>\r\n	In this study, there is a significant correlation between Bcl-2 expression and early tumour stage of the disease in which the tumours tend to be better differentiated. A relative reduction of Bcl-2 expression in the late stages of breast carcinoma suggests a downregulation of Bcl-2 with tumour development. This finding indicates that Bcl-2 expression in early breast carcinoma is associated with a good prognosis. Our results are in accordance with a study done by Leek et al. (1994) who had reported that loss of Bcl-2 expression was associated with poor outcome and that its expression was associated with improved survival (Berardo et al. 1998, Callagy et al. 2006). Bcl-2 has also been shown to be a powerful independent prognostic marker for patients with early breast cancer independent of adjuvant therapy (Dawson et al. 2010).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The present study demonstrated an inverse relationship between p53 and &nbsp;Bcl-2 protein expressions indicating that overexpression of mutant p53 induces downregulation of Bcl-2 in breast carcinogenesis. Our results also demonstrated that Bcl-2 is expressed in the early stages of breast cancer. The combined Bcl-2 overexpression and loss of p53 expression are useful markers in predicting good prognostic outcome in patients with breast cancer.</div>\r\n', 1),
(929, 924, '<p>\r\n	&nbsp;</p>\r\n<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Primary healthcare plays a central role in healthcare systems worldwide. It is the first level of care accessed by communities in the national healthcare system, providing not only curative treatment but also health promotion, preventive and rehabilitative cares. In Malaysia, primary care services are provided by public and private primary care clinics. The public primary care sector is mainly funded by the Ministry of Health Malaysia (MOH). In 2011, there were about three times more registered private primary care clinics compared to the public primary care clinics; 6,589 registered private clinics versus 1,973 public clinics (Ministry of Health Malaysia 2012). Apart from the private and MOH-funded public primary care sectors, public universities offering medical courses have their own hospitals and primary care clinics. These facilities are funded by the Ministry of Higher Education Malaysia and function as centres to carry out undergraduate and/or postgraduate training, clinical services to nearby communities, as well as medical research.</div>\r\n<div>\r\n	Primary healthcare places great emphasis on patient-centred care, promoting providers to be more responsive in identifying and meeting patients&rsquo; needs, preferences and expectations. Generally, patients&rsquo; experience and perception that meet or exceed their expectations has been the concept of patient satisfaction as proposed by Parasuraman et al. (1985). However, this concept focuses on functional (process) dimension of a service. This dimension is described as how a service is delivered such as provider&rsquo;s reliability, responsiveness, assurance and empathy, physical facilities, as well as equipment. However, the concept of patient satisfaction has evolved to becoming more complex that includes technical aspects of the service (i.e. service outcomes and provider&rsquo;s competence) (Wan Rashid &amp; Jusoff 2009, Mpinga &amp; Chastonay 2011).</div>\r\n<div>\r\n	Assessing patient satisfaction is of paramount importance as it predicts patients&rsquo; utilisation of health-care services and compliance to the treatment (Al-Eisa et al. 2005) as well as continuity of care (Donahue et al. 2005). Satisfied patients also tend to take an active role in their own healthcare (Donabedian 1997). Moreover, assessment of patient satisfaction with the services provided has been used as a measure of the service quality, allowing for identification of quality gaps and implementation of improvement strategies (Wan Rashid &amp; Jusoff 2009, Mpinga &amp; Chastonay 2011).</div>\r\n<div>\r\n	Studies on patient satisfaction has been extensively done worldwide since 1980&rsquo;s (Evans et al. 2007, Mpinga &amp; Chastonay 2011), while in Malaysia, a number of hospitals started to perform such studies a decade later (Haliza et al. 2005). However, the findings were not published (Haliza et al. 2005). Over the past 10 years, studies on patient satisfaction have been increasingly carried out in Malaysia. These studies were done in various healthcare settings i.e. in hospitals and primary care clinics, both public and private (Raja Jamaluddin 2001, Haliza et al. 2003, Yunus et al. 2004, Haliza et al. 2005, Noor Hazilah &amp; Nooi 2009, Mohd Suki et al. 2009, Nor Hayati et al. 2010, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011, Suki et al. 2011, Noor Hazilah 2012), various specialities (e.g. emergency department, dental, paediatrics, and obstetrics and gynaecology) (Pitalok &amp; Rizal 2006, Saiboon et al. 2008, Aniza et al. 2011, John et al. 2011) and groups of patients (e.g. patients with osteoporosis, cleft palate, and orthognathic problems) (Siow et al. 2002, Noor &amp; Musa 2007, Lai et al. 2010).</div>\r\n<div>\r\n	In Malaysia, most of the studies done in the primary care settings have showed high levels of patient satisfaction (78.8-92.3%), except a study by Haliza et al. (2005) that was carried out between 1996 and 1997 (Raja Jamaluddin 2001, Yunus et al. 2004, Haliza et al. 2005, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011). Haliza et al. (2005) demonstrated an overall patient satisfaction rate of 3.8% and 19.4% among patients who visited the public clinics and the private clinics respectively. Perhaps, re-organisation of our primary healthcare system over the past 10 years has resulted in an increase in patient<span style="font-size: 1.2em; line-height: 1.7em;">satisfaction as demonstrated by recent studies (Yunus et al. 2004, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011).</span></div>\r\n<div>\r\n	Factors that influence patients satisfaction can be generally divided into: (a) patient-related factors (e.g. age, race, gender and socio-economic status), (b) medical personnel-related factors (e.g. technical skills, time spent during a visit, communication, interpersonal relationship, and manners), and (c) system-related factors (e.g. accessibility, facilities, appointments, referrals, and continuity of care) (Hutchinson 1993, Haliza et al. 2003, Yunus et al. 2004, Haliza et al. 2005, Thiedke 2007, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011). Hence, these factors are commonly being assessed in determining patient satisfaction. Many studies have shown that interpersonal relationship has greater influence on patient general satisfaction as compared to other influencing factors, such as system-related factors (e.g. accessibility, facilities, appointments, referrals, and continuity of care) (Mendoza et al. 2001, Gadallah et al. 2003).</div>\r\n<div>\r\n	Eventhough a substantial number of studies on patient satisfaction have been carried out in Malaysia, studies examining patient satisfaction in the university-based primary care clinics are still scarce. Furthermore, recent advances in the public primary healthcare and expected enhancement in patient expectation with service provision, prompt further studies to assess present patient satisfaction.Therefore, this study aimed to evaluate the level of patient satisfaction with services provided by the Universiti Kebangsaan Malaysia Medical Centre (UKMMC) primary care clinic, assessing both general patient satisfaction with the overall service as well as their satisfaction with doctors, nurses, accessibility, facilities and appointments.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a descriptive cross sectional study, carried out in the UKMMC primary care clinic from February to March 2011. A total of 317 patients were recruited through systematic random sampling. The sample size met the precision requirements (&alpha;) of 0.05 and confidence level of 95%. The proportion used in the sample size calculation was the one reported by Sharifa Ezat et al. (2010), which was 86.1%.</div>\r\n<div>\r\n	In this study, all patients aged 18 years and above, who had attended the clinic for at least one visit in the past, were eligible for participation. However, those who required emergency care or could not read English or Bahasa Malaysia (BM) were excluded. During the study period, every third patient who registered at the registration counter was approached. Those who met the inclusion criteria were invited as participants. However, if the patient did not fulfil the inclusion criteria, the next patient registered at the counter would be selected. This study received approval from the Research and Ethics Committee of the Universiti Kebangsaan Malaysia (FF-001-2011).</div>\r\n<div>\r\n	A validated self-administered Patient Satisfaction Questionnaire (PSQ-46) was used as it was specifically designed to measure patient satisfaction with primary care services (Grogan et al. 1995). PSQ-46 was developed in the United Kingdom based on a qualitative study on factors affecting satisfaction with the primary care services. This study revealed 10 dimensions as follows: &ldquo;doctor information getting, doctor information giving, doctor social skills, doctor competence, doctor time pressure, access (access to doctor, receptionist, emergency service and out-of hours service), facilities (condition of waiting room and building), nurses (communication skills and interpersonal manners), receptionists/appointment (convenience and availability), and general satisfaction&rdquo; (Grogan et al. 1995). The questionnaire assesses not only the functional dimension but also the technical dimension of the healthcare service. It includes measuring patients&rsquo; perception on doctor&rsquo;s competency in performing thorough assessment and investigations, as well as prescribing treatment. Permission was obtained to use the PSQ-46 from the original author, Professor Sarah Grogan.</div>\r\n<div>\r\n	PSQ-46 comprises of 46 items with a five-point Likert &lsquo;agree-disagree&rsquo; scale. These items are categorised into: general patient satisfaction (six items) and satisfaction with five different subscales including doctors (20 items), nurses (four items), accessibility (eight items), facilities (four items) and appointment (four items). This categorisation allows us to identify patient dissatisfaction in specific areas of the service (doctors, nurses, accessibility, facilities and appointment).</div>\r\n<div>\r\n	PSQ-46 assesses almost similar components of patient satisfaction measured by Haliza et al. (2005) and Aniza &amp; Suhaila (2011). These questionnaires were Patient Satisfaction Questionnaire II (PSQ II) and Patient Satisfaction Questionnaire III (PSQ III), both originally developed from the one by Ware and Snyder (1975). However, PSQ-46 does not have dimensions of &lsquo;service charges&rsquo; and &lsquo;continuity of care&rsquo;. SERVQUAL is another instrument commonly used by many studies that examine consumer satisfaction with a broad range of services provided by various organisations (Ilhaamie 2010). The dimensions assessed by SERVQUAL are generic and focus on functional service quality. Many studies on patient satisfaction in Malaysia had used SERVQUAL but it was modified to meet the objectives of the studies (Yunus et al. 2004, Mohd Suki et al. 2009, Sharifa Ezat et al. 2010, Nor Hayati et al. 2010, Suki et al. 2011, John et al. 2011).</div>\r\n<div>\r\n	The original English version of PSQ-46 was translated into BM through forward and backward translation by two translators. Then, 10 participants examined the construct of BM-version questionnaire through face validation process. Subsequently, a pilot study was done on 30 respondents from the clinic. Reliability analysis of the pilot study showed a Cronbach&rsquo;s alpha coefficient of 0.882.</div>\r\n<div>\r\n	The collected data were analyzed by using SPSS version 19.0. The findings of each subscale were categorized into satisfied and not satisfied. The median of each subscale ([(maximum score &ndash; minimum score)&divide;2] + [minimum score]) was used as the cut-off point. All findings to demonstrate association between variables were analyzed using Chi square test. However, when the assumptions for the Chi square test were not met, Fisher&rsquo;s Exact test was used. Statistical significance was set at p&lt;0.05.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 317 respondents participated in this study, with a response rate of 77%. Majority of the respondents were female (64.7%, 205/317), Malays (67.2%, 213/317), aged between 35-64 years old (65.0%, 206/317), and received secondary education and above (89.6%, 284/317) (Table 1). More than half of the respondents (56.8%, 180/317) belonged to the working group, 33.4% (106/317) of the respondents worked in the government service and the other 26.2% (83/317) worked in the private sector. Approximately, two thirds of the respondents came from lower and middle-income families.</div>\r\n<div>\r\n	Majority of the respondents in this study were generally satisfied with the overall services (93.1%, 295/317), doctors (96.5%, 306/317), nurses (82.0%, 260/317), appointment (86.1%, 273/317), and accessibility (68.1%, 216/317) (Figure 1). However, only 35.6% of the respondents were satisfied with facilities.</div>\r\n<div>\r\n	There was no significant association between the socio-demographic characteristics and patients&rsquo; general satisfaction (Table 2). Analysis of each subscale demonstrated significant association between each of the subscales and general patient satisfaction (Table 3). Overall, majority of the respondents who were satisfied with doctors (95.1%, 291/306), nurses (97.7%, 254/260), accessibility of the service (97.7%, 211/216) or appointment system (98.2%, 111/113) were satisfied with the services generally. However, most of those who were dissatisfied with nurses (71.9%, 41/57), accessibility of the service (83.2%, 84/101), facilities (90.2%, 184/204) or appointment system (75.0%, 33/44) admitted to be generally satisfied as well. Only a third of the respondents who were not satisfied with the doctors were generally pleased and most of these respondents confessed their dissatisfaction with the overall services.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Overall, patients who attended UKMMC primary care clinic during the study period were generally satisfied with the services. This high level of satisfaction was similar with previous local studies done in primary care settings (Raja Jamaluddin 2001, Yunus et al. 2004, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011). Only one study by Haliza et al. (2005), which was done between 1996 and 1997, showed extremely low levels of satisfaction with the services among patients in public (overall patient satisfaction rate of 3.8%) and private clinics (overall patient satisfaction rate of 19.4%). Improvements most probably held by the government in both sectors contributed to this increase in patient satisfaction over the past decade (Ariff &amp; Teng 2002). Another reason to explain the high level of satisfaction found in this study is the &lsquo;generosity factor&rsquo; of patients giving good score when answering a questionnaire (Kasalova 1995).&not;This phenomenon can be explained by their fear of repercussion for giving genuine personal opinion, which may be construed as negative, despite reassurances that their future treatment would not be compromised (Angelopoulou et al. 1998, Coyle &amp; William 1999, Noor Hazilah &amp; Nooi 2009).</div>\r\n<div>\r\n	In our study, majority of the respondents expressed satisfaction with most of the areas of the service assessed (doctors, nurses, accessibility and appointment). The respondents were particularly satisfied with the doctors in terms of their communication skills, interpersonal manners and technical competence as assessed by the PSQ-46. This may be because the majority of the doctors in the UKMMC primary care clinics were registrars in training and family medicine specialists who optimally worked to realise the principles of family medicine, such as patient-centred and evidence-based care. None of other local studies done in the primary care settings specifically examined the quality of the treating doctors as holistic as our study. Nevertheless, Haliza et al. (2003, 2005) had assessed the technical quality of the doctors involved in their study, in which more than two-thirds of the participants were satisfied with their doctors&rsquo; technical quality. Other local studies only appraised the communication skills, interpersonal manners and/or technical quality of the clinic staff in general (Yunus et al. 2004, Sharifa Ezat et al. 2010, Aniza &amp; Suhaila 2011). All of these studies showed high levels of patient satisfaction in the aspects mentioned above, except a study by Sharifa Ezat et al. (2010). In this study, about 60% of patients in both private and public primary care clinics were dissatisfied with the staff&rsquo;s professionalism and caring attitude.</div>\r\n<div>\r\n	A significant number of our respondents, who were satisfied with the doctors, were also generally satisfied. Likewise, those who were not satisfied with the doctors expressed dissatisfaction with the general service. Similar association was not seen with all other subscales suggesting that doctors could be the main contributing factor in determining patient satisfaction. This finding was in parallel with other studies, which demonstrated that physician-related factors were the most important influence of patient satisfaction (Gadallah et al. 2003, Thiedke 2007, Thung &amp; Chan 2009).</div>\r\n<div>\r\n	In contrast, among the five subscales, two-thirds of our respondents were dissatisfied with the facilities. This is probably due to the overcrowded waiting area with limited space and seats in the UKMMC primary care clinic. However, Haliza et al. (2005) reported high levels of patient satisfaction with the clinics&rsquo; environment and the basic amenities.</div>\r\n<div>\r\n	Nonetheless, we found that respondents who were dissatisfied with the facilities, accessibility and appointment were still satisfied generally. This is probably due to low expectation and non-complaining nature of the respondents. They understood the limitations of government clinics (Noor Hazilah &amp; Nooi 2009) and were more accepting towards the shortcomings in the services. Moreover, patients paid less medical fees as compared to private clinics; hence they were less demanding (Noor Hazilah &amp; Nooi 2009). The majority of our respondents belong to low and lower-middle family income groups, they might not have the luxury of seeking medical care in private settings. Even though the association between socioeconomic status of the respondents and their satisfaction with the service was not statistically significant in our study, Yunus et al. (2004) did show a significant correlation between these two factors.</div>\r\n<div>\r\n	The findings in our study cannot be generalised to represent the quality of other primary care clinics in this country, as there were a few limitations. First of all, our study was conducted in a university-based primary care clinic, which is run by a group of trained specialists and postgraduates. The service providers are different from those in the public primary care clinics who are mainly medical officers without postgraduate qualifications. In addition, the public primary care clinics usually operate with limited manpower. The doctor-patient ratio is different compared to other clinics as well. Furthermore, not all aspects of the services were evaluated in this study, for example pharmacy, waiting time, continuity of care, drug prescription and laboratory investigations. These factors were found to be significantly influencing patient satisfaction in other studies (Haliza et al. 2003, Morgan et al. 2004, Gadallah et al. 2003, Thiedke 2007, Noor Hazilah &amp; Nooi 2009).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Overall, majority of respondents were satisfied with the services. However, many respondents perceived that clinic facilities were substandard. Nonetheless, majority who were not satisfied with accessibility, facilities and appointment were satisfied generally, which is probably due to low expectation and the non-complaining nature of the respondents. Despite this, we still need to improve areas deemed to be substandard. Thus, effective interventions towards improving facilities will further increase patient satisfaction and hence enhance the quality of care provided by the UKMMC primary care clinic. At the same time, continuous improvement should be made on all the evaluated aspects to ensure advancement in the quality of care. This strategy is concurrent with the increasing demands and expectations of patients in the coming decades. Further studies can also be extended to include other primary health care clinics to give broader perspectives regarding quality of primary health care services in Malaysia.</div>\r\n', 1),
(944, 939, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">INTRODUCTION</span></p>\r\n<div>\r\n	Red cell alloimmunisation in pregnancy is an immune disorder due to an incompatibility between maternal and fetal red blood cell antigens (Roback et al. 2008). Haemorrhage of fetal red cells into the maternal circulation can occur spontaneously during pregnancy or with trauma, amniocentesis, cordocentesis, abortion, and other manipulations.&nbsp;</div>\r\n<div>\r\n	The development of the alloantibody is due to the anamnestic response (Roback et al. 2008). With the first exposure to the foreign antigen, the B lymphocyte clones are invoked which will result in a moderate production of IgM or IgG antibodies. It is the secondary exposure that elicits a more rapid production of large quantities of significant IgG-class antibody which can cross the placenta. Destruction of the fetal red cells occurs when the maternal antibody binds to the fetal red cell antigen, causing attachment to the Fc receptor of the macrophages in the spleen of the fetus and leads to a condition called haemolytic disease of the fetus and newborn (HDFN) (Denise 2005 &amp; Roback et al. 2008).The newborn infants with HDFN present with severe anaemia and jaundice. As an example, in a Rh-negative mother who is exposed to the fetal Rh-positive red cells, the maternal immune system will produce anti-D antibodies. In a subsequent pregnancy, these anti-D antibodies will cross the placenta into the fetal circulation and result in haemolysis.</div>\r\n<div>\r\n	Approximately 1% of pregnant women are found to have clinically significant red cell antibodies and there are more than 50 red cell antigens that can elicit antibodies that have been found to cause HDFN (Howard et al. 1998 &amp; Moise 2000). Of these, the commonest specificity is anti-D, although universal introduction of routine antenatal anti-D prophylaxis has reduced this sensitisation rate (Howard et al. 1998 &amp; Moise 2000). However, with the introduction of antenatal anti-D prophylaxis, there has been a significant rise in positive antibody screening results, due to the detection of the prophylactic anti-D (Howard et al. 1998). Other alloimmune antibodies that can result in HDFN include other Rh (E, e, C, c), Kell (K and k), Duffy (Fya and Fyb), Kidd (Jka and Jkb), and MNSs (M, N, S and s) (Moise 2000 &amp; Roback et al. 2008). Multiparous women and mothers with bad obstetric history (history of abortion, neonatal death and stillbirth) have been found to have higher chance of alloimmunisation due to multiple exposures to the incriminating foreign antigens (Patel et al. 2009 &amp; Shanwell et al. 1999).</div>\r\n<div>\r\n	According to guidelines issued by the British Committee for Standards in Haematology, it has been suggested that ABO and Rh as well as the antibody screening be carried out during the antenatal follow up to detect the presence of maternal alloantibody. Therefore, this study aimed to look at the prevalence of development of significant alloantibodies in pregnant women in this centre and to identify the common alloantibodies encountered. The knowledge will help reduce the risk of complication of haemolytic disease of the fetus and newborn and further emphasise whether routine antibody screening during antenatal check-up is justified.&nbsp;</div>\r\n<div>\r\n	MATERIALS and METHODS</div>\r\n<div>\r\n	This cross-sectional study was done on women who had antenatal follow-up at the antenatal clinic of a tertiary hospital. The study was approved by the Institutional Review Board and Ethics Committee. Women with previous history of blood transfusion, women with autoimmune hemolytic anaemia and those on antibiotics were excluded from the study. The patients were randomly selected during the period of study from January 2012 to April 2012. A total of 150 patients&rsquo; blood samples were subjected to pre-transfusion testing which included ABO- and Rh-grouping and antibody screening. Data on age, gravida, parity, past medical and obstetrical history were taken. For each patient, 5 mls of blood was taken in two EDTA tubes. The pretransfusion testing were done following the American Association of Blood Bank standard which consists of identification of the recipient and blood sample collected, ABO and Rh D typing, and antibody screening. The antibody screening was done using indirect antiglobulin test, performed by gel agglutination technique using Diamed gel cards. For antibody screening, 3-cell screening panels (Diamed ID-Dia cell) were used. The positive results were further identified for antibody specificity by the Diamed ID-Dia Panel (11 cell panel), including the use of enzyme treated cells (papain) at 37˚C and anti human globulin (AHG) phase.</div>\r\n<div>\r\n	RESULTs</div>\r\n<div>\r\n	During the study period, only two patients were found to have positive antibody screening out of a total of 150 patients&rsquo; samples, giving an incidence of 1.3%. The two positive samples were identified as anti-C and anti-D. Table 1 shows gravidity versus antibody formation. In our study, 56 samples were primigravidae (37.3%), 34 women were gravida 2 (22.6%), 28 women were gravida 3 (18.7%), 19 women were gravida 4 (12.7%) and 13 were of gravida 5 or more (8.7%). No alloimmunisation was found among primigravida, gravida 4 and gravida 5 or more. The two positive samples were from gravida 2 and gravida 3 with incidences of 2.9% and 3.6% respectively. The incidence of antibody production was less in normal cases (0.87%) than in bad obstetric history cases (2.86%) as expected (Table 2).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Red cell alloimmunisation can result in significant consequences to pregnancy outcomes. The production of clinically significant anti-D in an Rh negative mother has been well established in causing severe haemolytic disease of the fetus and newborn. However, with the current practice of pretransfusion testing which includes ABO- and Rh-grouping, the possible clinical problems associated with anti-D can be anticipated and abated early with the use of Rh0 (D) immunoglobulin or better known as Rhogam. &nbsp;Nevertheless, there are more than 50 red cell antigens which are able to elicit antibody production in HDFN cases (Howard et al. 1998 &amp; Moise 2000).</div>\r\n<div>\r\n	Red cell alloimmunisation may also pose a significant problem in blood transfusion practice. It may result in difficulty in finding compatible blood for transfusion and could result in grave consequences especially during emergency. The frequency of patients being alloimmunised to non-D antibodies has been rising in the past decades (Lobo et al. 2006 &amp; Yousuf et al. 2012). Another study, Yousuf et al. (2012) found that the prevalence of positive antibody screening among transfusion recipients was 0.76% (184/24,263) of pretransfusion samples. In this study, the most frequent single alloantibody was anti-Mia (29.1%, 48/165), followed by anti-E (18.6%, 30/165). Antibody screening, which has been implemented in many developed countries, help identify and specify antibodies in potential recipients and thus antigen-negative blood can be prepared in the event of blood transfusion.</div>\r\n<div>\r\n	In pregnancy, however, the incidence of red cell alloimmunisation is 1% to 2% (Howard et al. 1998; Moise 2000; Solola et al. 1983 &amp; Weinstein 1982). Lee et al. (2003) reported the prevalence of clinically significant antibodies among Chinese pregnant women to be 0.27%. Different ethnic backgrounds may be a confounding factor that influences the prevalence of alloantibody production in pregnant women. Our study which was done on a multiracial population, demonstrated a prevalence of 1.3% (2/150, one of chinese and one of malay ethnicity). Nevertheless, due to the small sample size, a bigger study is needed to improve the power of the study and to differentiate between ethnic groups in this country.&nbsp;</div>\r\n<div>\r\n	Red cell alloantibodies detected in two (1.3%) patients were identified as anti-C and anti-D from a gravida 2 (2.9%) and gravida 3 (3.6%) respectively. &nbsp;None of the primigravidae was alloimmunised. &nbsp;Each pregnancy increases a woman&rsquo;s risk of fetomaternal circulation leak, and therefore increases the chance of alloimmunisation.&nbsp;</div>\r\n<div>\r\n	With the same understanding, there would be less chance of antibody production in women with normal pregnancy than in those with bad obstetric history. Our data shows the incidence for alloimmunisation of 0.87% (1/115) in women with normal obstetric history and 2.86% (1/35) in those with bad obstetric history. In this study, the gravida 2 had a history of an early neonatal death and she was positive for anti-C. The other patient, who is a gravida 3 at 11 weeks gestation, is a Rh negative mother with a previous normal obstetric history. &nbsp;She was found to be positive for anti-D, with a titre of 1:32 (2IU/ml), most likely from the Rhogam which was administered to her a few days earlier due to threatened miscarriage. Passive anti-D can be detected for 12 weeks or more after anti-D prophylaxis has been given (BCSH 2006). Nevertheless, in this case active immunisation still could not be excluded as it is difficult to differentiate anti-D immunoglobulin from an immune anti-D.</div>\r\n<div>\r\n	From the relatively small sample the prevalence for alloimmunisation of 1.3% among pregnant women in our centre was in accordance with other studies (Howard et al. 1998; Moise 2000; Solola et al. 1983 &amp; Weinstein 1982). The antibodies detected were anti-C and anti-D, which belong to the Rh group and the incidence of red cell alloimmunisation were higher in women with bad obstetric history. Nevertheless, a bigger sample size over a longer study period is needed for better demonstration of the true prevalence.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The prevalence of red cell alloimmunisation among pregnant women in this centre is low, similar to other population studies. Nevertheless, it is important that red cell antibody screening be included as part of the antenatal pretransfusion testing in accordance with the current international guidelines (BCSH 2006), so that pregnancies at risk of HDFN can be identified and appropriate management provided. Where laboratory facilities are limited or cost is a factor, selective screening of the patients especially those with previous bad obstetric history and history of blood transfusion should be implemented.</div>\r\n<div>\r\n	ACKNOWLEDGMENTS&nbsp;</div>\r\n<div>\r\n	The authors wish to thank the staff of the Blood Bank Unit and Laboratory C staff (Department of Laboratory and Diagnostic Services) for their contribution and the Universiti Kebangsaan Malaysia Fundamental Grant (FF-059-2012) for financial support.&nbsp;</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(946, 941, '<p>\r\n	<span style="font-size: 12px; line-height: normal;">INTRODUCTION</span></p>\r\n<div>\r\n	Dengue fever is a common mosquito- borne viral infection in the tropical and subtropical regions affecting millions of people yearly. It is a potentially life-threatening condition due to haemorrhaegic complications. Ocular manifestation is not common and was previously considered rare. Kalpoor et al. (2006) reported that the most common clinical finding in patients with dengue fever was petechial type of subconjunctival haemorrhages (Kalpoor et al. 2006). Posterior segment involvements were vascular tortuosity and retinal haemorrhages, sparing the macula region (Kalpoor et al. 2006). Other common findings are blot retinal haemorrhages and retinal vessel tortuosity. The patho-mechanism of ocular involvement is not clearly understood, and treatment options for ocular complications of dengue fever are not established (Chang et al. 2007). Management of dengue-related ophthalmic complications are largely conservative. Steroid has been used in selected cases with extensive vasculitis or exudative retinal detachment (Chan et al. 2006). The visual prognosis is good, but most patients have residual scotoma with good Snellen visual acuity. This particular case presented to the ophthalmology unit prior to diagnosis of dengue fever, and highlights the importance and possibility of ocular manifestation of this condition to both the ophthalmologists and physicians. Furthermore, it illustrates that conservative management in a patient not suitable for steroid therapy is still effective, and should be considered in selected cases, especially when steroid side effects is the main issue.</div>\r\n<div>\r\n	case report</div>\r\n<div>\r\n	A 37 year old lady presented with a one day history of central scotoma in both eyes. She had persistent fever for one week and noted skin rash three days previously. She was otherwise well and was breastfeeding at the time of presentation.&nbsp;</div>\r\n<div>\r\n	Ocular examination showed reduced visual acuity of 20/200 in both eyes. Anterior segment examination was unremarkable. The most obvious findings on funduscopy were bilateral macular oedema with small, discreet subretinal deposits and blot retinal haemorrhages. Optic discs and retinal vessels were normal (Figure 1). There were no other peripheral retinal lesions in both eyes.&nbsp;</div>\r\n<div>\r\n	Laboratory investigations were performed including full blood count, dengue and Chikungkunya serology. A diagnosis of dengue fever was made based on low platelet count of 106x109/L, an elevated haematocrit level of 38.7% and positive dengue IgM. Serology for chikungkunya was negative. Toxoplasmosis and syphilis serology were negative and tuberculosis was ruled out by negative chest X-ray and Mantoux test. Amsler chart test showed a large area of central scotoma on both eyes. Optical coherence tomography (OCT) demonstrated bilateral subretinal fluid collections (Figure 2). Patient opted not to proceed with fluorescein angiography as she is exclusively breastfeeding at that time.</div>\r\n<div>\r\n	Intravenous fluid (normal saline 0.3%) was instituted, and the patient recovered from dengue fever without major haemodynamic derangement. She regained her baseline visual acuity at six weeks after her first presentation. However, she still perceived areas of scotoma in her peripheral visual field at one year follow up.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Dengue fever is an endemic health issue which raises public health concern due to its dramatic increase in the incidence in recent years. Severe dengue fever known as dengue haemorrhagic fever is life threatening. In endemic areas, dengue is diagnosed by clinical symptoms of fever, typical rash and thrombocytopaenia. However confirmatory diagnosis is based on IgG and IgM serology for antibody detection, (Buchy et al. 2006) and real- time polymerase chain reaction for dengue RNA serotypes (Lanciotti et al. 1992).</div>\r\n<div>\r\n	Ocular involvements in association with dengue fever have been reported, mainly of reduction in visual acuity and visual image distortion secondary to macular changes. The visual metamorphosia is thought to arise from subretinal fluid accumulation (Su et al. 2007). The exact patho-mechanism of dengue-related ocular complications is unknown, but it may be attributed to an underlying immunological reaction, which may lead to inflammatory response in retinal tissue and retinal vascular systems. Visual disturbance can be debilitating especially when it occurs bilaterally, and in a young mother such as the present case.</div>\r\n<div>\r\n	The appearance of ocular symptoms and onset of dengue fever may vary from 2 to 15 days. Typically, ocular manifestations of dengue fever occur one week following the onset of illness, when other symptoms are improving, and correspond to development of immune-complex and antibody formation (Su et al. 2007). This immune-mediated ocular reactions to systemic infection corresponds with production of autoantibodies and deposition of immune complex (Lim et al. 2004). A study utilizing electrophysiology by Chia et al. (2006) found that middle and outer retina are more severely affected compared to the more-spared inner retina at the foveal region (Chia &nbsp;et al. 2006). It has been reported that most patients recover from their visual functions, however occlusive vasculitis from dengue-related vasculopathy may result in permanent visual loss due to development of retinal ischaemia. Our case is unusual as patient presented with ocular symptoms as a presentation of dengue fever, although she noticed the appearance of skin rash three days prior to that. To the best of our knowledge, this is the first report of a patient who presented with dengue maculopathy before the diagnosis of dengue fever was made.</div>\r\n<div>\r\n	The recovery of dengue maculopathy typically occurs four to six weeks from the onset, but residual scotoma and metamorphopsia is often permanent. Visual acuity typically improves with resolution of macular oedema and recovery of the dengue fever, however, residual paracentral scotoma is common and may be permanent. It is postulated that the immune-complex deposition at the retinal pigment epithelium and choroid cause structural and permanent damage, (Lanciotti et al. 1992) hence the residual scotoma. Affected patients may need to be counseled regarding the long term effects of residual scotoma and follow up may be necessary in cases of non-resolving subretinal fluid. Teoh et al. (2010) found that different patterns of retinal fluid collection such as foveolitis, diffuse retinal thickening and cystoids macular oedema are useful predictors of visual outcome in dengue-related maculopathy (Teoh et al. 2010). The same study concluded that diffuse type of retinal thickening, independent of extent of oedema predicts shortest period of persistence of scotoma. Optical coherence tomography is also a useful tool in assessing the progression or improvements and as an aide in counseling patients.</div>\r\n<div>\r\n	Regarding the management of dengue fever, there is no specific treatment for dengue related maculopathy, and recovery in visual acuity commonly occurs without immunosuppressive treatment. A conservative approach is important in certain cases such as patients who are breastfeeding, pregnant or have contraindications to steroid. However, monitoring and patients&rsquo; education are equally important. In selected cases of extensive inflammatory reaction to retinal tissue and retinal vessels resulting in exudative retinal detachment or retinal ischaemia, immunosuppressive agents may be necessary. Various treatment regime with immunomodulating agents such as intravenous methylprednisolone, oral steroids, intravitreal triamcinolone and immunoglobulin have been tried (Bascal et al. 2007). Bascal and colleagues observed that treatment of dengue maculopathy with steroids are more beneficial in patients with foveolitis and subretinal fluid accumulation, compared to retinal vaso-occlusive cases (Bascal et al. 2007).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Dengue is an endemic viral fever with ocular manifestations that may affect useful vision. Dengue- related maculopathy improves with conservative management, but residual visual disturbance may be permanent. Optical coherence tomography is a useful tool for diagnosis and monitoring of patients with maculopathy. Fluorescein angiogram is important to confirm presence of ischaemic retina or vasculitis, but may not always be the method of choice for breastfeeding or pregnant patients.</div>\r\n', 1),
(950, 945, '<p>\r\n	&nbsp;</p>\r\n<div>\r\n	BACKGROUND</div>\r\n<div>\r\n	Aneuploidy is one of the most frequent genetic abnormalities found in cancer (French et al. 2003; Pihan &amp; Doxsey 2003; Upender et al. 2003; Jonsson et al. 2008; Espadinha et al. 2009). It was known to be the cause of cancer over a century ago, based on Hansemann&rsquo;s observations of asymmetric mitoses in epithelial cancers (Hansemann 1890), which was further proven in 1964, when Boveri showed evidence that aneuploidy in developing sea urchin embryos generates abnormal phenotypes (Boveri 1964).&nbsp;</div>\r\n<div>\r\n	Aneuploidy, resulting from full or partial aneusomies in which the copy number of entire chromosomes or chromosomal subregions is altered, has been proposed to be an early and genetically destabilizing force in cancer development (French et al. 2003). Increasing aneuploidy is associated with cancer progression and considered as a sign of malignancy, but the molecular mechanisms involved in aneuploidy in cancer remains undefined in the majority of human cancers, particularly in thyroid cancer (French et al. 2003; Banito et al. 2007).&nbsp;</div>\r\n<div>\r\n	The distribution of aneuploid cells within tumour cells is not uniform, indicating that thyroid cancers may be clonally heterogeneous, which is consistent with the notion that thyroid cancer cells may have chromosomal instability (Ouyang et al. 2002). Possibly due to the varying chromosome composition, aneuploid cancer cells usually exhibit a wide spectrum of clinical aggressiveness (Isaka et al. 2003). These aneuploid cells could continue to segregate asymmetrically every time they divide in the &ldquo;chromosome error propagation&rdquo; process (Carlson et al. 2000). This may explain the association of aneuploid cells in cancer patients with poor prognosis (Ouyang et al. 2002; Barril et al. 2000). There are reports suggesting that follicular carcinomas without PPAR&gamma; protein overexpression are more prone to develop distant metastasis, to invade locally, to present with poorly differentiated areas and to persist after surgery, suggesting an association between PPAR&gamma; protein underexpression with a less differentiated phenotype (Espadinha et al. 2009; Marques et al. 2004).<span class="Apple-tab-span" style="white-space:pre"> </span></div>\r\n<div>\r\n	Previous studies on thyroid neoplasms reported common aneusomy of chromosome 3, 7, 10, 12 and 17 (Barril et al. 2000; Criado et al. 1995; Belge et al. 1998). Aneusomy of chromosome 7 was also reported in bladder (Wolman et al. 2007) and prostate cancer (Alcaraz et al. 1994), while aneusomy of chromosome 17 was reported in squamous cell carcinoma of the vulva (Carlson et al. 2000), carcinoma of the breast (Wang et al. 2002) and lung (Nakamura et al. 2003). French and co-workers reported 3p25 aneusomy in 29% follicular carcinoma of the thyroid (French et al. 2003). The 3p25 region is associated with PPAR&gamma; gene, which was widely investigated for its involvement with PAX8 gene in differentiating follicular thyroid carcinoma (FTC) from its benign counterpart (French et al. 2003; Marques et al. 2004; Kroll et al. 2000; Nikiforova et al. 2002; Dwight et al. 2003; Nikiforova et al. 2003; Cheung et al. 2003; Hibi et al. 2004; Lui et al. 2004; Sahin et al. 2005; Nikiforov 2010; Chia et al. 2010).</div>\r\n<div>\r\n	Although rare in papillary thyroid carcinomas (PTCs), aneuploidy is a common feature of follicular thyroid adenomas (FTAs) and carcinomas (FTCs) (Espadinha et al. 2009; Banito et al. 2007; Ouyang et al. 2002). Aneuploidy is observed in about 57% FTCs, 10-25% FTAs and 10-22% multinodular hyperplasia (Espadinha et al. 2009; Banito et al. 2007). Besides FTAs and FTCs, one study reported that aneuploidy was also detected in a subset of FTC that shows oncocytic features (Dettori et al. 2003). Some studies have reported that there is a tendency of an increased aneuploidy rate, generally from follicular adenomas to follicular carcinomas and from minimally invasive to widely invasive follicular carcinomas (Grant et al. 1990; Oyama et al. 1994).&nbsp;</div>\r\n<div>\r\n	World Health Organization (WHO) has categorized Hurthle cell neoplasms (HCN) as a variant of follicular neoplasms, as both neoplasms possess distinct features (Kroll 2004). Among the distinct features that differentiate the HCNs from FTN are the abnormal accumulation of mitochondria in the cytoplasm (Tallini et al. 1999), distinct morphologic features (Kroll 2004), frequent resistance to radio-iodine therapy (Stojadinovic et al. 2002), frequently detected RET rearrangements (Chiappetta et al. 2002) and lack of RAS mutations and PPAR&gamma; rearrangements in HCN (Nikiforova et al. 2003).&nbsp;</div>\r\n<div>\r\n	In this paper, we present three cases of follicular thyroid neoplasms (two FTC and one HCA) with 3p25 aneusomy detected by FISH analysis without PPAR&gamma; protein overexpression.&nbsp;</div>\r\n<div>\r\n	CASE PRESENTATION</div>\r\n<div>\r\n	Clinicopathological Findings</div>\r\n<div>\r\n	Case 1 (FC-03)</div>\r\n<div>\r\n	A 30 year-old lady presented to the surgery clinic of Universiti Kebangsaan Malaysia Medical Centre (UKMMC) with a right thyroid swelling. Fine-needle aspiration cytology (FNAC) yielded cellular smears comprising of thyroid epithelial cells arranged in sheets and follicular pattern. The cells show generally round nuclei with scanty inspissated colloid. The overall features were reported as consistent with a follicular neoplasm. Total thyroidectomy was performed.</div>\r\n<div>\r\n	Sections from the mass showed a partially encapsulated tumor consisting of neoplastic cells arranged in solid sheets as well as follicular pattern. Collections of inspissated colloid with multifocal areas of haemorrhage and necrosis were also present within the tumor. Generally, the cells display uniform round to oval nuclei with evenly distributed chromatin. Few mitotic figures were evident. A focal area of minimal capsular invasion was noted. However, presence of vascular invasion was not seen. There was no evidence of malignancy in the left thyroid gland and the isthmus. A diagnosis of FTC (minimally invasive) was made.</div>\r\n<div>\r\n	Case 2 (FC-19)</div>\r\n<div>\r\n	A 60 year-old lady presented to the surgery clinic, Putrajaya Hospital with a left thyroid swelling. FNA showed cellular smears comprising of neoplastic epithelial cells arranged in microfollicles with scanty to no colloid. Neoplastic cells display nuclear crowding and overlapping. A cytological diagnosis of follicular neoplasm was made.&nbsp;</div>\r\n<div>\r\n	Total thyroidectomy was performed. Sections from the right lobe and isthmus showed thyroid follicles of varying sizes intersected in areas by fibrous septae. Some of the thyroid follicles appeared dilated. There were areas of haemorrhage and oedema in the intervening stroma. Aggregates of foam cells were also present.&nbsp;</div>\r\n<div>\r\n	Sections from the left thyroid lobe showed tumour composed of closely packed follicles and trabeculae. The tumour cells were cuboidal with hyperchromatic nuclei. Areas of calcification and haemorrhage were noted but mitotic activity was inconspicuous. In some areas, capsular invasions were seen but no vascular invasion was present. A diagnosis of FTC was made.</div>\r\n<div>\r\n	Case 3 (FA-18)</div>\r\n<div>\r\n	A 29 year-old man presented to the surgery clinic, UKMMC with a well-defined neck mass which moved with swallowing. The mass measured 3cm in diameter.&nbsp;</div>\r\n<div>\r\n	FNAC yielded scanty material. Smears showed a few thyroid epithelial cells arranged in microfollicles as well as singly dispersed cells displaying abundant granular cytoplasm with mild anisonucleosis. Cytological features were reported as suspicious of a follicular neoplasm, Hurtle Cell type.&nbsp;</div>\r\n<div>\r\n	The excised specimen consists of two nodular lesions, each measuring 3.5 x 2 x 1.5 cm and 1 x 1.1 x 1.5 cm. The larger nodule revealed a partially encapsulated mass composed of small colloid filled follicles lined by cells with abundant eosinophilic cytoplasm with mild nuclear pleomorphism. Some of the cells showed a trabecular pattern of arrangement. Sections from the smaller nodule showed variable sized colloid filled follicles lined by cuboidal epithelium. The nodules were separated by fibrous bands. No capsular invasion was seen.&nbsp;</div>\r\n<div>\r\n	A histological diagnosis of follicular adenoma of the thyroid (Hurthle cell type) was made.&nbsp;</div>\r\n<div>\r\n	Cytogenetic Findings</div>\r\n<div>\r\n	FISH analysis was performed on the three follicular thyroid neoplasms, i.e. one HCA and two FTCs. Briefly, 3&mu;m thick formalin-fixed paraffin-embedded (FFPE) tissue sections were cut from each of the cases. The sections were deparaffinised in 1&times; SkipDewax (Insitus Biotechnologies, New Mexico, USA) at 80oC for 30 minutes and digested with 1.5mg/ml pepsin (Sigma-Aldrich Co., St. Louis, USA) at 37&deg;C for 1 hour. The FFPE tissue sections were hybridized with an in-house PAX8 (green)/ PPAR&gamma; (orange) dual colour extra-signal bacterial artificial chromosome-fluorescence in-situ hybridization (BAC-FISH) fusion probe assay (Chia et al. 2010) overnight at 37&deg;C. After hybridization, the slides were washed, counterstained, analysed and documented using an epifluorescence microscope system (Applied Spectral Imaging System, Germany). Normal nuclei showed a two orange and two green signal pattern.</div>\r\n<div>\r\n	3p25 aneusomy was detected in all three follicular thyroid neoplasms. Three orange and two green signal pattern was seen in the trisomy nuclei of one of the FTCs (FC-03). The other case of FTC (FC-19) and the HCA (FA-18) showed four orange and two green signal patterns, indicating 3p25 tetrasomy (Figure 1). All three cases displayed the respective signal patterns in more than 50% of the 200 nuclei analysed. No PAX8/PPAR&gamma; translocation was observed.</div>\r\n<div>\r\n	Immunohistochemical Findings</div>\r\n<div>\r\n	Immunohistochemistry was performed on FFPE tissue sections of all three follicular neoplasms using the monoclonal antibody PPAR&gamma; (Santa Cruz Biotechnology, Santa Cruz, CA). Antigen retrieval was performed in 10mM citrate buffer (pH 9.9) at 95&deg;C for 40 minutes before incubating the slides with primary antibody (1:30) for 30 minutes. The slides were then incubated with secondary antibody (ChemMate Dako Envision, HRP rabbit/mouse secondary antibody) and stained with DAB chromogen (DAKO) according to the manufacturer&rsquo;s instruction. No PPAR&gamma; overexpression was detected in all three follicular thyroid neoplasms, i.e. two FTCs and one HCA.&nbsp;</div>\r\n<div>\r\n	DISCUSSION&nbsp;</div>\r\n<div>\r\n	In this paper, we report three cases of 3p25 aneusomy associated with low PPAR&gamma; protein expression in a Hurthle cell adenoma and two follicular carcinomas of the thyroid. It would be interesting to see whether similar findings have been reported by other authors, as these observations were seen in both benign and malignant lesions of two different entities of follicular thyroid neoplasms.&nbsp;</div>\r\n<div>\r\n	Aneusomy is often associated with a poor prognosis in cancer patients (Barril et al. 2000) and is considered as an adverse factor in thyroid carcinoma, although the cause of it remained undefined (Banito et al. 2007). Based on the study by Marques et al. (2004), clinically aggressive tumours had lower PPAR&gamma; protein expression than less aggressive carcinomas (Marques et al. 2004). This is further supported by the fact that trisomies 7 and 12 in PTCs (which are rarely aneuploid) and FTCs showed a poor clinical outcome (Barril et al. 2000). There are also reports on the association between polysomy (aneusomy or aneuploidy) and high-grade or late stage cancers (Placer et al. 2005; Panani et al. 2004).&nbsp;</div>\r\n<div>\r\n	Although there were studies suggesting that aneuploidy was associated with genetic instability of many cancers (Banito et al. 2007; Barril et al. 2000; Wolman et al. 2007), it is still not clear why aneuploidy is such a highly variable prognostic marker for different tumours. Lengauer et al. (1998) reported that aneuploidy may not necessarily present with chromosomal instability (Lengauer et al. 1998). While chromosome losses or gains during cell division may trigger apoptosis in normal cells, tumour cells may be protected from apoptosis through a pre-existing mutation (Ouyang et al. 2002). Along this process, the rate of acquisition of chromosomal changes may be normal, and thus aneuploidy develops in the absence of chromosomal instability (Ouyang et al. 2002). Meanwhile, Isaka and co-workers (2003) postulated that the wide spectrum of clinical aggressiveness among different aneuploid cancers is due to the existence of different types of cancer aneuploidy created by various molecular defects in gene networks governing the cell cycle, recombination, the repair of double-stranded deoxyribonucleic acid (DNA) breaks and the mitotic checkpoints (Isaka et al. 2003).</div>\r\n<div>\r\n	None of the three follicular thyroid neoplasms analysed using the in-house PAX8 (green)/ PPAR&gamma; (orange) dual color extra-signal BAC-FISH fusion probe assay (Chia et al. 2010) had both 3p25 aneusomy and PAX8/PPAR&gamma; rearrangement, which indicates the involvement of separate independent genetic pathways in early follicular thyroid tumorigenesis. French et al. (2003) suggested either the PAX8/PPAR&gamma; rearrangement or 3p25 aneuploidy can occur in early follicular carcinomas (French et al. 2003), which is similar to the detection of either RAS mutation or PPAR&gamma; rearrangement in FTCs (Tallini et al. 1998; Koenig 2010) as well as the detection of either RET rearrangements or NTRK1 rearrangement in PTCs (Pierotti &nbsp;2001).&nbsp;</div>\r\n<div>\r\n	Previous cytogenetic studies have reported a high prevalence of trisomies and tetraploidies in FTN (Belge et al. 1998; Hemmer et al. 1998; Roque et al. 1999). We observed presence of aneuploidy in all three cases, both benign and malignant follicular lesions, which is comparable with other previous reports (Banito et al. 2007; Barril et al. 2000; Hemmer et al. 1998; Johannessen et al. 1982). The presence of 3p25 aneusomy in both follicular adenoma and carcinoma of the thyroid support a stepwise adenoma to carcinoma sequence, or indicate the presence of carcinoma in situ. The appearance of 3p25 aneusomy in a HCA reported in this paper further suggests that gain of the 3p25 region is an early event in the development of follicular thyroid neoplasms.&nbsp;</div>\r\n<div>\r\n	In conclusion, the presence of the aneusomy in both cases of HCA and FTC indicates that there could be a biological continuum between the two neoplasms, and chromosomal gains might play an important role in the pathogenesis of these two types of neoplasms (Chia et al. 2010). Although there has been considerable speculation as to whether Hurthle cell tumours may be clinically and histologically distinct from other follicular thyroid neoplasms, the detection of 3p25 aneusomy in the case of HCA alongside with two FTCs suggests that Hurthle cell neoplasms may be considered a separate follicular thyroid tumour entity but shared the same genetic abnormality in the development of follicular thyroid tumour.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to thank Mr. Clarence Ko Ching Huat for his technical advice in the preparation of FISH probe. This project was funded by a research grant from the Malaysian Ministry of Science, Technology and Innovation (MOSTI); grant number 06-02-02-0054 EA250 and approved by UKM ethics committee (UKM FF-031-2005).</div>\r\n', 1);
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(954, 949, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Introduction</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Inflammation is part of normal host response to infection and injury. Although inflammation helps clear infection, and along with repair, makes wound healing possible, both inflammation and repair have considerable potential to cause harm. If the agents causing inflammation persist for prolonged period of time, acute inflammation can worsen and becomes chronic inflammation.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Chronic inflammation can result from viral or microbial infection, environmental antigen, autoimmune reaction or persistent activation of inflammatory molecules and is associated with increased risk of human cancer at various sites (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_15" title="Hussain, 2003 #2">Hussain et al. 2003</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_4" title="Bartsch, 2006 #1">Bartsch &amp; Nair 2006</a>). Chronic inflammation processes increase oxidative and nitrasative stress and lipid peroxidation by inducing oxidant-generating enzymes such as NADPH oxidase, inducible nitric oxide synthase (iNOS) thereby producing excess of diverse reactive oxygen and nitrogen species (ROS and RNS). ROS, RNS and lipid peroxidation products (LPO) are able to cause damage to cell, protein, mRNA, DNA and lipid in both nuclear and mitochondrial (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_25" title="Ohshima, 2003 #3">Ohshima et al. 2003</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_26" title="Sawa, 2006 #4">Sawa &amp; Ohshima 2006</a>). Lipid peroxides (LPO) such as malondialdehyde (MDA), can also modify DNA bases to form various exocyclic products (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_11" title="Chung, 1996 #5">Chung et al. 1996</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_2" title="Bartsch, 1999 #6">Bartsch 1999</a>), which in turn can modulate signaling molecules (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_33" title="West, 2006 #7">West &amp; Marnett 2006</a>), and alter the function of enzymes and proteins involved in inflammation and carcinogenesis (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_24" title="Ohshima, 2003 #8">Ohshima 2003</a>)such as cyclooxygenase-2 (COX-2) and iNOS.</p>\r\n<p>\r\n	Chronic inflammation is abnormal and the excessive ROS, RNS and LPO produced, facilitate carcinogenesis and other chronic diseases by producing various types of DNA damage, overproduction of proinflammatory cytokines and reducing apoptotic rates of damage cells (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_15" title="Hussain, 2003 #2">Hussain et al. 2003</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_3" title="Bartsch, 2005 #9">Bartsch &amp; Nair 2005</a>).&shy;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp; Several medicinal herbs have been shown to inhibit inflammation as well as antioxidant activity. The antioxidant activity can further protect from oxidative and nitrosative stress damages induced by inflammation process. Compounds from fenugreek spice (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_22" title="Liu, 2012 #10">Liu et al. 2012</a>), and polyphenols from green tea(<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_30" title="Tipoe, 2007 #11">Tipoe et al. 2007</a>)have been found to exhibit anti-inflammatory and antioxidant activities. <em>Centella asiatica</em>, a perennial creeper growing abundantly in moist areas and distributed widely in tropical and subtropical countries, has long history of medicinal uses in Ayurvedic and Chinese traditional medicines for treatment of wounds, ulcer and leprosy (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_8" title="Brinkhaus, 2000 #13">Brinkhaus et al. 2000</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_10" title="Cheng, 2000 #12">Cheng &amp; Koo 2000</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_27" title="Shetty, 2006 #14">Shetty et al. 2006</a>). It is locally known as <em>pegaga</em>. The bioactive pentacyclic triterpenoid compounds of centella asiatica, Asiaticoside and madecassoside, have been widely studied, and reported to have various biological&nbsp; properties such as wound healing (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_28" title="Shukla, 1999 #15">Shukla et al. 1999</a>), ulcer healing (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_17" title="Jin, 2004 #16">Jin et al. 2004</a>), anti-apoptosis (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_7" title="Bian, 2008 #17">Bian et al. 2008</a>), and also immunomodulatary (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_16" title="Jayathirtha, 2004 #18">Jayathirtha &amp; Mishra 2004</a>).</p>\r\n<p>\r\n	Various methods have been described to prepare centella asiatica extract. However, there is a lack of comparative data on the best method for extracting these bioactive compounds in terms of antioxidant and anti-inflammatory properties. In this study, we compared three different extraction methods for <em>C.asiatica</em> using ethanol, methanol and water and then evaluated the extracts in terms of the phenolic, asiaticosside and madecassoside contents, antioxidant and anti-inflammatory activities.&nbsp;</p>\r\n<p>\r\n	<strong>Materials and Methods</strong></p>\r\n<p>\r\n	<em>Plant material</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Centella asiatica</em>was bought from a local market in Kuala Lumpur and sample was deposited at UKM herbarium with voucher number UKMB29984. The whole plant was washed thoroughly with tap water, and dried at 40<sup>0</sup>C&nbsp; until constant weight and coarsely grounded. Pure standards of asiaticoside and madecassoside were obtained from Biopurify (Chengdu, China).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Extracts Preparation</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Sohxlet extraction was used for the preparation of methanol and ethanol extracts. 50g of <em>Centella asiatica</em> coarse plant powder was extracted with 1 litre of either methanol or ethanol for 24 hours and then filtered to collect the extracts. The extracts were then concentrated under vacuum and excess solvent were removed using rotary evaporator.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; For the preparation of aqueous extract, 50 g of coarse plant powder in 1 litre of distilled water was boiled for 1 hour and the extract filtered. Extract were then freeze-dried.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Determination of asiaticoside and madecassoside content</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The level of asiaticoside and madecassoside in all CA extracts were determined using HPLC. 1mg of ethanol or methanol CA extracts were dissolved in 1ml of methanol. As for the aqueous extract, 1mg of extract was dissolved in ultra pure water. Sample extracts were filtered through 0.2&micro;m filter. A volume of 20&micro;l was injected using an autosampler into the HPLC (Shimadzu, Japan) equipped with a diode array detector. The solvent used for gradient elution were 1% ortophosphoric acid and acetonitrile. The concentration of 1% ortophosphoric acid was set at 5% for the first 5 minutes and then decreased to 80%, 50% and 20% for the next 10, 5 and 7 minutes respectively. The concentration of the 1% ortophosphoric acid was then increased to 50%, 80% and 95% for 5 minutes for each concentration. The analytical column used was ACE C18 with packing material of 5&micro;m particle size at a flowrate of 1.0ml/min at room temperature. Samples were read at 210nm wavelength. Pure standards of asiaticoside and madecassoside concentration range (15&micro;g/ml to 500&micro;g/ml) were used to calibrate standard curve and retention times.&nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Total phenolic content assay</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Folin-Ciocalteu reagent (diluted 10-fold) was used to determine the total phenolic content of all CA extracts (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_32" title="Velioglu, 1998 #33">Velioglu et al. 1998</a>). 100 &micro;l of each sample was mixed with 0.75 ml of Folin-Ciocalteu reagent and allowed to stand at 22<sup>o</sup>C for 5 minutes before adding 0.75 ml &nbsp;sodium bicarbonate (60 g/L). The mixture was incubated at 22<sup>o</sup>C for 90 minutes and then read at 725 nm wavelength. Rutin was used as standard for the construction of calibration curve. The assay was carried out in triplicates.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>2,2-diphenyl-1-picrylhydrazyl (DPPH) assay</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The radical scavenging activity of each extract was determined using<em>2,2-diphenyl-1-picrylhydrazyl</em> (DPPH) assay as described byUllah et al, 2009 (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_31" title="Ullah, 2009 #34">Ullah et al. 2009</a>). 0.25ml aliquots of each of the CA extracts or bioactive compounds (10-1000&micro;g/ml) were added to 0.75ml of freshly prepared ethanol DPPH solution (0.02mM). The optical density changes were measured at 517nm after 30 minutes. Blanks were used to remove the influence of colour of the samples. Ethanolic solution of DPPH was used as negative control. Butylated hydroxytoluene (BHT) and ascorbic acid were used as positive controls in the same concentration range (50-1000 &micro;g/ml) as were used for the samples. Free radical scavenging activity was expressed as inhibition percentage and was calculated using the following formula :</p>\r\n<p>\r\n	% free radical scavenging activity = <img border="0" height="29" src="///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image002.gif" width="162" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Ferric Reducing Antioxidant Power (FRAP) assay</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Working FRAP reagent was prepared by mixing 25 ml of acetate buffer pH 3.6, 25 ml of&nbsp; TPTZ (2,4,6-tripyridyl-s-triazine) solution, 2.5 ml of FeCl<sub>3</sub>.6H<sub>2</sub>0 solution and 3 ml of distilled water (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_6" title="Benzie, 1996 #35">Benzie &amp; Strain 1996</a>). The reaction of mixture of aliqout of CA extracts and bioactive compounds &nbsp;(25 &micro;l) with working FRAP reagent (175 &micro;l) was monitored for up to 4 minutes and the optical density changes were read at 593 nm. FRAP values were obtained by comparing the absorbance change at 593 nm in test reaction mixtures with those containing ferrous ion in known concentration using FeSO<sub>4</sub>.7H<sub>2</sub>O solution.&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>COX enzymes assay</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Anti-inflammatory activity of CA extracts and bioactive compounds was evaluated by its ability to inhibit the inflammatory pathway enzyme, cyclo-oxygenase (COX) which catalyses the production of PGE<sub>2</sub>. This was determined using COX Inhibitor Screening Assay Kit ( Cayman Chemical ACE, USA). 20&micro;l of extract solutions (30&micro;g/ml) or&nbsp; bioactive compounds &nbsp;(5&micro;g/ml) were added to the COX-1 and COX-2 tubes respectively. The mixtures were incubated for 10 minutes at 37<sup>o</sup>C. The reaction was initiated by the addition of 10&micro;l of arachidonic acid to all the test tubes. The tubes were shaken vigorously and allowed to stand for another 2 minutes at 37<sup>o</sup>C. After that, 50 &micro;l of 1 M HCl was added to each test tube to stop the reaction. Test tubes were removed from the water bath and 100&micro;l of the saturated stannous chloride solution was added to each test tube. The tubes were then shaken vigorously before leaving them to stand for 5 minutes at room temperature. The prostaglandin E<sub>2</sub> (<em>PGE<sub>2</sub></em>) &nbsp;produced were then quantified using a commercial Prostaglandin E<sub>2</sub> Monoclonal-EIA Kit ( Cayman Chemical ACE, USA). &nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Suppression of TPA-induced prostaglandin E2 production</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The ability of the extracts, asiaticoside and madecassoside to suppress TPA-induced inflammation was evaluated by measuring the production of PGE2 which is produced during inflammatory process. Fibroblast cells were incubated with &nbsp;TPA and either of the extracts, asiaticosside or madecassoside for 24 hours. The amount of PGE2 produced was measured using the Prostaglandin E<sub>2</sub> Monoclonal-EIA Kit ( Cayman Chemical ACE, USA).</p>\r\n<p>\r\n	&nbsp;Cells (5 x 10<sup>3</sup>) were seeded in <em>Dulbecco modified Eagle&rsquo;s medium</em> (DMEM) supplemented with 10% fetal calf serum (FCS) in 96-well plate and were incubated in a humidified atmosphere with 5% CO<sub>2</sub>, 37<sup>o</sup>C, for 24 hours. Cells were then incubated with &nbsp;12-<em>O</em>-tetradecanoylphorbol-13-acetate (TPA) and either of the extracts, asiaticosside and madecassoside for a further 24 hours. The supernatant from the tissue culture was then collected and quantified by EIA procedure.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Results and Discussion</strong></p>\r\n<p>\r\n	<em>Total Phenolic and HPLC analysis</em></p>\r\n<p>\r\n	The level of total phenolic constituents, asiaticoside and madecassoside in the three different extracts are shown in Table 1.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The results showed that ethanol is the most effective solvent in extracting phenolic compounds as well as the bioactive compounds, asiaticoside and madecassoside. The total concentration of phenolic compounds extracted &nbsp;in this study for the three different extraction solvents was higher than that previously reported by Zainol et al 2003 (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_34" title="Zainol, 2003 #20">Zainol et al. 2003</a>).The levels of asiaticoside and madecassoside in both ethanol and methanol extracts of CA were in agreement with previous study which reported that values of triterpenic acid sugar esters including asiaticoside and madecassoside of 1-8% of plant material (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_18" title="Kartnig, 1992 #21">Kartnig &amp; Hoffmann-Bohm 1992</a>)and higher than that reported by Jayathirta and Mishra, 2004 who reported a yield of &nbsp;only 0.18% of asiaticoside (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_16" title="Jayathirtha, 2004 #18">Jayathirtha &amp; Mishra 2004</a>).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; However the yield of asiaticoside (0.28% ) using aqueous extraction produced lower yield than that reported by Jin et al 2004 (4% ). The low yield of asiaticoside and madecassoside in our aqueous extract could be due to the use of high temperature during extraction of plant materials. It has been reported that, high temperature would decrease the concentration of asiaticoside, and madecassoside (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_19" title="Kormin, 2005 #22">Kormin 2005</a>). Distribution of asiaticoside and madecassoside in C.<em>asiatica</em> plant were also organ specific, with the highest concentration found in leaves (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_1" title="Aziz, 2007 #23">Aziz et al. 2007</a>). Variations in asiaticoside content also has been associated with plant origin, Centella asiatica growing at higher altitude contained more asiaticoside than plants from lower altitude (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_12" title="Das, 1991 #24">Das &amp; Mallick 1991</a>).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Antioxidant activity by DPPH and FRAP assay</em></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	The 2,2-diphenyl-1-picrylhydrazyl (DPPH) assay was used to determine the antioxidant activity of CA extracts based on their ability to trap unpaired electrons of DPPH. Extracts and compounds that could decolorize DPPH solution (purple) to yellow suggest their antioxidant potential. The DPPH scavenging activity of CA extracts in the concentration range of 50 - 1000 &micro;g/ml are shown in (Figure 1).</p>\r\n<p align="center">\r\n	&nbsp;</p>\r\n<p>\r\n	Among CA extracts, the aqueous extract showed the strongest antioxidant activity at 1000 &micro;g/ml reaching a 79.4 % of DPPH radicals scavenging activity compared to methanol extract and ethanol extract with 77.3 % and 65.7 % respectively, at the same concentration level. &nbsp;However both asiaticoside and madecassoside did not show any detectable DPPH scavenging activity, hence data is not shown.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The ferric-reduction ability of plasma (FRAP) assay also enable the evaluation of antioxidant potential based on the ability to reduce ferric ion (Fe3+) to ferrous ion (Fe2+).The antioxidant potential is shown as FRAP reagent decolorizes from purple to blue, as a result of &nbsp;the formation of ferrous-tripyridyltriazine (TPTZ) complex. The FRAP values of CA extracts in the concentration range of 500 &micro;g/ml - 2000 &micro;g/ml are shown in (Table 2). At all &nbsp;concentrations, all CA extracts reduced the ferric-diTPTZ less than ascorbic acid and tocotrienol-rich fraction (TRF) that were used as positive controls. Among the extracts, aqueous extract showed slightly better ferric reducing ability than ethanol and methanol extracts at all concentration tested. Consistent with DPPH assay, both asiaticoside and madecassoside did not show appreciable ferric reducing activity hence data not shown.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Despite having the lowest total phenolic content among the CA extracts, aqueous extract possessed the strongest free radical scavenging activity as determined by the DPPH assay. Based on previous study, the antioxidant activity of polar extract could be due to the presence of compounds with free hydroxyls, such as flavonoids (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_23" title="Mensor, 2001 #25">Mensor et al. 2001</a>). Levels of flavonoids in the aqueous extract of CA could be higher compared to ethanol and methanol extract due to being extracted in most polar water, compared to less polar ethanol and methanol solvent. Flavonoids make good antioxidant agent since they possess a number of free hydroxyls which can scavenge free radicals (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_9" title="Cao, 1997 #27">Cao et al. 1997</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_5" title="Basile, 2005 #26">Basile et al. 2005</a>). In contrast to our findings, previous study on CA extraction has shown that ethanol extract had the highest antioxidant activity when compared with aqueous extract (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_14" title="Hamid, 2002 #28">Hamid et al. 2002</a>). However the differences might be due to different methods of extraction and assays used to measure the antioxidant activity. The results also suggest that the antioxidant activity of CA resides in water soluble compounds. This is also in agreement with several reports that high polarity solvents are effective in extracting natural antioxidants (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_13" title="Duh, 1992 #29">Duh et al. 1992</a>; <a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_29" title="Tian, 1994 #30">Tian &amp; White 1994</a>).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<em>Anti-inflammatory activity</em></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The CA extracts and bioactive compounds were tested for COX-1 and COX-2 enzyme inhibitory activity. At 30&micro;g/ml, the ethanol, methanol and aqueous extracts inhibited COX-1 and COX-2 enzymes by 97.84% and 97.91%, 98.18% and 96.16%, 83.06% and 72.10%, respectively. Asiaticoside and madecassoside at 5&micro;g/ml concentration inhibited COX-1 and COX-2 enzymes by 98.08% and 83.67%, and 91.57% and 89.79%, respectively. Aspirin were used as positive control at 5&micro;mg/ml concentration and inhibited COX-1 and COX-2 by 80.98% and 88.86%, respectively (Fig.2). Among CA extracts, ethanol and methanol extracts showed stronger inhibition of &nbsp;COX-1 and -2 than the aqueous extract. Asiaticoside and madecassoside inhibitory activity of COX-1 and COX-2 enzymes were similar to commercial NSAIDs, aspirin.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p align="center">\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The anti-inflammatory activity of CA extracts and compounds were further studied using TPA-induced inflammation in human fibroblasts. Treatments with 30&micro;g/ml concentration of ethanol, methanol and aqueous extracts of CA on the TPA-induced fibroblast cells produced significantly less PGE<sub>2</sub> compared to control (TPA alone). Treatment of the cells with asiaticoside and madecassoside at 5 &micro;g/ml also significantly reduced the TPA-induced formation of PGE<sub>2</sub> (Fig.3).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Figure 3 also showed that ethanol and methanol extracts of CA were more potent than aqueous extract in suppressing the TPA-induced formation of PGE2 while Figure 2 showed that these extracts were better inhibitors of the COX &nbsp;activity.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Bioactive compounds of CA, asiaticoside and madecassoside suppressed PGE<sub>2 </sub>formation at similar level. Both compounds were also found to inhibit TPA-induced inflammation in fibroblasts by modulating COX activity as shown by their inhibitory effects in COX-1 and -2 enzymes assay. Our findings also agree with previous work on collagen-induced arthritis mice which treatment with asiaticoside were reported to inhibit inflammation by reducing the expression of COX-2 and inflammatory cytokines (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_21" title="Li, 2007 #31">Li et al. 2007</a>). Previous work on similar model of arthritis, have found that oral administration of madecassoside to reduce the expression of COX-2 and PGE<sub>2</sub> production, but did not change the expression of COX-1 (<a href="///D:/ina/UKKMC/Medicine%20&amp;amp;%20Health/1.%20To%20reviewer(s)/MS151/Musalmah%20Mazlan%207Jan2013.doc#_ENREF_20" title="Li, 2009 #32">Li et al. 2009</a>).</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The more potent anti-inflammatory activity of ethanol and methanol extracts of CA in regressing inflammation in TPA-induced fibroblasts than the aqueous extract could be due to higher content of phenolics, and /or bioactive compounds asiaticoside and madecassoside in both ethanol and methanol CA extracts compared to the aqueous extract. Asiaticoside and madecassoside, both have shown to have anti-inflammatory activity, and these compounds may react synergistically with each other or with other phenolic constituents to give CA its anti-inflammatory activity. &nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;<strong>Conclusion</strong></p>\r\n<p>\r\n	The present study demonstrated that hydrophobic solvent extraction of <em>Centella asiatica</em> yielded better yields in terms on phenolic, asiaticosside and madecassoside contents and that this is associated with better anti-inflammatory potential. &nbsp;</p>\r\n<p>\r\n	<strong>Acknowledgments</strong></p>\r\n<p>\r\n	The authors would like to thank the Ministry of Science, Technology and Environment of Malaysia for supporting the study (Grant No. 02-01-02-SF0525) and Universiti Kebangsaan Malaysia for the laboratory facilities.</p>\r\n', 1),
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(964, 959, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>PHOTOPROTECTIVE MEASURES OF SELECTED SUNSCREENS AND ITS ANTIOXIDANTS ADJUVANTION EFFECTS IN MALAYSIAN CLIMATE</strong></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Running title: photoprotective measures of sunscreens</p>\r\n<p>\r\n	Keywords: sunscreen, sun protection factor (SPF), ultraviolet A Protection Factor (UVA PF),</p>\r\n<p>\r\n	photostability, antioxidant.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>ABSTRAK</strong></p>\r\n<p>\r\n	Industri penjagaan kulit Malaysia sedang berkembang maju dengan memperlihatkan peningkatan bilangan krim pelindung cahaya matahari yang dihasilkan setiap tahun. Kekurangan peraturan dan kawalan dalam aspek penjagaan kesihatan kulit menyebabkan ketidaktentuan kualiti krim pelindung cahaya matahari yang dihasilkan. Objektif kajian ini adalah untuk menentukan keberkesanan dan keselamatan krim pelindung cahaya matahari tempatan berbanding dengan krim dari luar negara Malaysia serta kesan penambahan antioksidan asli ke dalam krim-krim tersebut. Tiga krim tempatan dan tiga krim luar negara telah dikaji dengan menggunakan kaedah COLIPA yang dimodifikasi untuk menentukan faktor pencegahan ultraviolet A in vitro (UVAPF) 2009. Sampel krim disapu pada plat polimetilmetakrilat (PMMA) yang kasar di mana penyerapan cahaya dibaca dengan spektrofotometer sebelum dan selepas dua jam pendedahan kepada radiasi cahaya untuk menentukan faktor pencegahan cahaya (SPF), UVAPF dan fotostabiliti. Kaedah yang sama diulangi dengan menambahkan krim pekat vitamin C dan E yang terdapat dalam pasaran. Krim dari luar negara didapati mempunyai label SPF yang jitu (p=0.009) manakala krim tempatan adalah lebih stabil terhadap cahaya (p=0.003). Walau bagaimanapun, kedua-dua krim tidak mempunyai UVAPF yang cukup (p=0.471). Penambahan vitamin C meningkatkan nilai SPF (p=0.04) kedua-dua krim manakala vitamin E meningkatkan fotostabiliti (p=0.000) mereka. Penambahan kombinasi vitamin C dan E kepada krim pelindung cahaya matahari dengan menariknya tidak mengubah nilai SPF dan UVAPF berbanding&nbsp; dengan penambahan satu vitamin secara berasingan. Yang pentingnya, kombinasi vitamin menurunkan fotostabiliti (p=0.002) krim berbanding dengan penambahan vitamin E sahaja.&nbsp; Kesimpulannya, pelindungan terhadap cahaya terbaik dicapai melalui penggunaan krim pelindung cahaya bersama dengan krim yang mengandungi hanya salah satu vitamin C atau E sahaja.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>ABSTRACT</strong></p>\r\n<p>\r\n	Malaysia skincare industry is growing fast seeing vast number of new sunscreens had been produced annually. Inadequacy of skincare regulations and lacking in its enforcement, amongst others has resulted in the overrating of these sunscreens&rsquo; quality. The objectives of this study were to evaluate the efficacy and safety of the local and international sunscreens available in Malaysian market and to determine the augmentation effects of adding concentrated antioxidants into them. Three local and three international manufactured sunscreens were tested using a modified COLIPA method for the in vitro determination of Ultraviolet A protection factor (UVAPF) 2009. The cream samples were spread onto roughened polymethylmethacrylate (PMMA) plates where the absorbance profiles of the samples before and after two hours of sunlight irradiation were measured using a spectrophotometer for the determination of in vitro Sun Protection Factor (SPF), UVAPF and photostability. The same procedure was repeated on&nbsp; the sunscreens with addition of commercially available concentrated vitamin C and E creams. International branded sunscreens were found to have more accurate SPF labels (p=0.009) while local branded sunscreens were found to be more photostable (p=0.003). However, both&nbsp; sunscreens did not provide adequate UVAPF (p=0.471). Addition of vitamin C enhanced the SPF&nbsp; values (p= 0.04) of both groups of sunscreens while vitamin E enhanced their photostability (p=0.000). Interestingly, adding a combination of vitamins C and E&nbsp; into the sunscreens did not&nbsp; improve the SPF and UVAPF values as compared to the use of a single vitamin. More importantly, the combination of vitamins decreased the photostability (p=0.002) of sunscreens as compared to addition of vitamin E alone. In conclusion, photoprotection is best achieved when sunscreens are used together with creams containing either vitamin C or E alone.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>1. INTRODUCTION</strong></p>\r\n<p>\r\n	Once an unprotected skin is exposed to the sunlight, ultraviolet (UV) radiation which is absorbed by the skin chromophores interacts with oxygen molecules to generate reactive oxygen species (ROS) such as hydrogen peroxide, superoxide radical anion, hydroxyl radical and singlet oxygen. Eventually, these hazardous ROS damages the neighboring deoxyribonucleic acid (DNA), proteins, and lipids in the skin (Bastuji-Garin et al, 2002). Therefore, prolonged and repeated exposures to sunlight would cause skin problems such as photodamage, photoaging and skin cancer (Gilchrest 2008).</p>\r\n<p>\r\n	Sunscreen is claimed to be the gold standard in protecting our skin from photodamage as it contains UV filters that may be present in the form of organic compounds, inorganic molecules or organic particulates (Seite S, 2000). When the skin is exposed to UV light, UV filters in sunscreen absorb, reflect and scatter the UV irradiation thus preventing it from reaching the skin. Antioxidants have been used as an adjuvant to sunscreens because of their ability to neutralize ROS generated by UV radiation (Seite S, 2000). Hence, a vast number of sunscreens containing antioxidants additives have been introduced in the market.</p>\r\n<p>\r\n	Sun Protection Factor (SPF) which measures the amount of protection against UVB (290-320 nm), is a multiple of UV irradiance required to cause erythema on a sunscreen applied skin as compared to a bare skin. A high SPF value indicates a better protection from sun burning of the skin. On the other hand, Ultraviolet A protection factor (UVAPF) measures the amount of protection to UVA (320-400 nm) radiation afforded by sunscreens where a high value indicate a better protection from persistent pigment darkening of the skin when exposed to sunlight (Diffey et al, 2000). In our search for sunscreen samples for this study, most sunscreens were found to have no information regarding this value.&nbsp;</p>\r\n<p>\r\n	Photostability is defined as the ability of a product to retain its integrity upon exposure to light. A product is considered photostable when the reduction of SPF and UVAPF values are less than 20% after exposure to light (Garoli et al, 2008).&nbsp; A research conducted by Herzog and Sommer (2000) found that chemical compounds in sunscreens that are unstable with exposure to sunlight may serve as another source of free radicals upon degradation.</p>\r\n<p>\r\n	Steven et al. (2010) found that antioxidant additives in some sunscreens had no effect in naturalizing ROS developed in photodamaged skin. Various reasons were postulated as the cause of this finding such as low antioxidant power, low biological activity, insufficient&nbsp; concentration of antioxidant as well as the inability of antioxidant to penetrate the skin. Thus, more effective methods and means of adding antioxidants to improve photoprotection are much needed.</p>\r\n<p>\r\n	Therefore, the aim of this study is to compare the photoprotective measures of some commercially available sunscreens in Malaysia. The efficacy and safety of international and local branded sunscreens is compared in terms of their SPF label accuracy, relative UVAPF level and their photostability.&nbsp; The adjuvant effects of adding concentrated vitamin C and E creams in improving photoprotection&nbsp; is also assessed.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>2. METHODOLOGY</strong></p>\r\n<p>\r\n	<strong>2.1 Sample size</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The sample size was calculated based on Mead&rsquo;s Resource Equations 1988. The error of degree of freedom was set in between 10-20. A total of three local and three international manufactured sunscreens were analysed in triplicates in this study, based on the European Cosmetic Products Trade Association (COLIPA) guideline for UVAPF 2009 determination (Table 1).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Table 1: Sample Size and Combination of the Test Samples</p>\r\n<table border="1" cellpadding="0" cellspacing="0" width="624">\r\n	<tbody>\r\n		<tr>\r\n			<td rowspan="2" style="width:109px;height:23px;">\r\n				<p>\r\n					Manufacture Place</p>\r\n			</td>\r\n			<td rowspan="2" style="width:132px;height:23px;">\r\n				<p>\r\n					Sunscreen Type</p>\r\n			</td>\r\n			<td colspan="7" style="width:383px;height:23px;">\r\n				<p>\r\n					Combination of&nbsp; Test Samples (unit)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:126px;height:23px;">\r\n				<p>\r\n					Sunscreens Only</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:23px;">\r\n				<p>\r\n					Vitamin C</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:23px;">\r\n				<p>\r\n					Vitamin E</p>\r\n			</td>\r\n			<td colspan="2" style="width:71px;height:23px;">\r\n				<p>\r\n					Vitamin C+E</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="3" style="width:109px;height:20px;">\r\n				<p>\r\n					International</p>\r\n			</td>\r\n			<td style="width:132px;height:20px;">\r\n				<p>\r\n					Sunscreen 1</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:20px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:20px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:20px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:20px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:132px;height:18px;">\r\n				<p>\r\n					Sunscreen 2</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:18px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:18px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:18px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:18px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:132px;height:17px;">\r\n				<p>\r\n					Sunscreen 3</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="3" style="width:109px;height:17px;">\r\n				<p>\r\n					Local</p>\r\n			</td>\r\n			<td style="width:132px;height:17px;">\r\n				<p>\r\n					Sunscreen 4</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:17px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:132px;height:22px;">\r\n				<p>\r\n					Sunscreen 5</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:22px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:22px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:22px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:22px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:132px;height:24px;">\r\n				<p>\r\n					Sunscreen 6</p>\r\n			</td>\r\n			<td colspan="2" style="width:126px;height:24px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:24px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:24px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n			<td style="width:70px;height:24px;">\r\n				<p>\r\n					3</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td colspan="2" style="width:241px;height:17px;">\r\n				<p>\r\n					Total</p>\r\n			</td>\r\n			<td style="width:126px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td colspan="2" style="width:96px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td colspan="2" style="width:90px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td colspan="2" style="width:71px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n		</tr>\r\n		<tr height="0">\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>2.2 Operational definitions</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The international and local sunscreens were selected from a range of commercially available sunscreens that were manufactured outside and in Malaysia respectively.&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>2.3 Inclusion criteria</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The selected sunscreens contained no antioxidants and had details of the country from where they were manufactured. The antioxidant creams contained mainly vitamins C or E as the active ingredient. Table 2 shows the details of sunscreens and antioxidant creams used in the experiment.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Table 2: Details for the Sunscreen and Antioxidant Creams</p>\r\n<p>\r\n	<strong><u>International Sunscreens</u></strong></p>\r\n<table border="0" cellpadding="0" cellspacing="0" style="width:624px;" width="624">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:120px;height:20px;">\r\n				<p>\r\n					Product</p>\r\n			</td>\r\n			<td style="width:108px;height:20px;">\r\n				<p>\r\n					SPF Label</p>\r\n			</td>\r\n			<td style="width:396px;height:20px;">\r\n				<p>\r\n					Major Ingredients</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:43px;">\r\n				<p>\r\n					Sunscreen 1</p>\r\n			</td>\r\n			<td style="width:108px;height:43px;">\r\n				<p>\r\n					50+</p>\r\n			</td>\r\n			<td style="width:396px;height:43px;">\r\n				<p>\r\n					Ethylhexyl Methoxycinnamate, Zinc Oxide, Diethylamino Hydroxybezoyl Hexyl Benzoate</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:23px;">\r\n				<p>\r\n					Sunscreen 2</p>\r\n			</td>\r\n			<td style="width:108px;height:23px;">\r\n				<p>\r\n					50+</p>\r\n			</td>\r\n			<td style="width:396px;height:23px;">\r\n				<p>\r\n					Zinc Oxide, Ethyhexyl Methocinnamate, CL 77891</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:42px;">\r\n				<p>\r\n					Sunscreen 3</p>\r\n			</td>\r\n			<td style="width:108px;height:42px;">\r\n				<p>\r\n					50+</p>\r\n			</td>\r\n			<td style="width:396px;height:42px;">\r\n				<p>\r\n					ButylMethoxydibenzoylmethane, Octocrylene, Benzophenone-3, Ethylhexyl Salicyclate</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong><u>Local Sunscreens</u></strong></p>\r\n<table border="0" cellpadding="0" cellspacing="0" style="width:624px;" width="624">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:120px;height:19px;">\r\n				<p>\r\n					Product</p>\r\n			</td>\r\n			<td style="width:108px;height:19px;">\r\n				<p>\r\n					SPF Label</p>\r\n			</td>\r\n			<td style="width:396px;height:19px;">\r\n				<p>\r\n					Major Ingredients</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:43px;">\r\n				<p>\r\n					Sunscreen 4</p>\r\n			</td>\r\n			<td style="width:108px;height:43px;">\r\n				<p>\r\n					20+</p>\r\n			</td>\r\n			<td style="width:396px;height:43px;">\r\n				<p>\r\n					Titanium Dioxide, Octyl Methoxyccinnamate, Butyl Methoxydibenzoylmethana,</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:29px;">\r\n				<p>\r\n					Sunscreen 5</p>\r\n			</td>\r\n			<td style="width:108px;height:29px;">\r\n				<p>\r\n					25</p>\r\n			</td>\r\n			<td style="width:396px;height:29px;">\r\n				<p>\r\n					Titanium Dioxide, Ethylhexyl Methoxycinnamate</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:29px;">\r\n				<p>\r\n					Sunscreen 6</p>\r\n			</td>\r\n			<td style="width:108px;height:29px;">\r\n				<p>\r\n					25</p>\r\n			</td>\r\n			<td style="width:396px;height:29px;">\r\n				<p>\r\n					Titanium Dioxide, Hydroxystearic Acid</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Antioxidant Creams</strong></p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:264px;height:17px;">\r\n				<p>\r\n					Product</p>\r\n			</td>\r\n			<td style="width:366px;height:17px;">\r\n				<p>\r\n					Major Ingredients</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:264px;height:38px;">\r\n				<p>\r\n					Vitamin E cream</p>\r\n				<p>\r\n					(Concentration: 10,000 iu/ 200 ml )</p>\r\n			</td>\r\n			<td style="width:366px;height:38px;">\r\n				<p>\r\n					Tocopheryl Acetate, Butylated hydroxytoluene (BHT)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:264px;height:45px;">\r\n				<p>\r\n					Vitamin C cream</p>\r\n				<p>\r\n					(Concentration: 50x)&nbsp;</p>\r\n			</td>\r\n			<td style="width:366px;height:45px;">\r\n				<p>\r\n					Glycyrrhiza Glabra Root Extract, Myrarciacia Dubia Fruit Juice, Malpighia Glabra Fruit Juice</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>2.4 Method</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Roughened polymethylmethacrylate (PMMA) plate with measurement of 40mm x 40mm x 2.5mm from NIC Advertising, Ampang, Selangor, Malaysia were used. An amount of 2mg/cm2 of sunscreens alone and 4mg/cm2 of the combinations of sunscreens with Vitamin C or/and E creams was added onto the plate with the aid of a weighing scale with variations &plusmn; 5% of weight. The test samples were applied onto the plate as multiple small droplets and then spread as even as possible. The test sample was then allowed to stabilize for 15 minutes in a dark, air conditioned room.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The absorbance profile of the test samples were measured by a two-beam UV/VIS Spectrophotometer Shimadzu UV-2450 spectrophotometer.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Polymethylmethacrylate (PMMA) plate covered with test samples was placed in a plate holder of the spectrophotometer. The UV absorbance of each test sample was measured from 290-400 nm at 1 nm intervals at five different places per plate. The data was read using UVProbe 2.33. The average of five readings was taken for analysis.</p>\r\n<p>\r\n	Since&nbsp; consumers are commonly advised to reapply sunscreens every two hours (Brian L. Diffey 2001),&nbsp; the test samples were also exposed under the sun for two hours (12:00 pm-14:00 pm). Table 3 shows the UV index of the day of the experiment provided by the Malaysian Meteorological Department, Petaling Jaya, Selangor, Malaysia. Hence, the average UV index for two hours of irradiation was 4.74.</p>\r\n<p align="center">\r\n	Table 3:UV Index at 31st of May 2012 by hour</p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					Time</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					10:23</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					10:58</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					11:32</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					12:07</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					12:42</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					13:14</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					13:26</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					14:04</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					14:44</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					15:22</p>\r\n			</td>\r\n			<td style="width:64px;height:29px;">\r\n				<p>\r\n					16:02</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					UV Index</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					4.55</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					5.91</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					3.84</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					4.44</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					4.33</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					6.96</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					4.08</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					4.16</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					7.27</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					5.43</p>\r\n			</td>\r\n			<td style="width:64px;height:37px;">\r\n				<p>\r\n					3.78</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The absorbance profile of the test samples after irradiation was taken again. All data we collected was analysed using SPSS ver.20</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>3. RESULTS</strong></p>\r\n<p>\r\n	Figure 1 shows examples of absorbance profiles of test samples. The shape and height of the curves varied depending on the type and concentration of UV filters used in the test samples. The in vitro SPF and UVAPF values as well as photostability were calculated based on the listed formulas.</p>\r\n<p align="center">\r\n	Figure 1: Example of Absorbance Profiles for Various Combinations of Test sample</p>\r\n<p>\r\n	&nbsp;<img height="207" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image002.gif" width="635" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Formulae Calculating In vitro SPF, In vitro UVAPF and Photostability</p>\r\n<p>\r\n	&nbsp;<img height="43" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image004.gif" width="283" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Where,</p>\r\n<p>\r\n	E (&lambda;)&nbsp;&nbsp;&nbsp; = Erythema Action Spectrum at wavelength &lambda;.</p>\r\n<p>\r\n	I (&lambda;) &nbsp;&nbsp;&nbsp; = Spectral Irradiance of UV source at wavelength &lambda;.</p>\r\n<p>\r\n	d &lambda;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp; = Wavelength step = 1nm</p>\r\n<p>\r\n	A (&lambda;)&nbsp;&nbsp;&nbsp;&nbsp; = Absorbance of the test samples at wavelength &lambda;.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<img height="57" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image006.gif" width="331" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	P (&lambda;)&nbsp;&nbsp;&nbsp; = Persistent Pigment Darkening Action Spectrum at wavelength &lambda;.</p>\r\n<p>\r\n	I (&lambda;) &nbsp;&nbsp;&nbsp; = Spectral Irradiance of UV source at wavelength &lambda;.</p>\r\n<p>\r\n	d &lambda;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp; = Wavelength step = 1nm</p>\r\n<p>\r\n	A (&lambda;)&nbsp;&nbsp;&nbsp;&nbsp; = Absorbance of the test samples at wavelength &lambda;.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;<img height="33" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image008.gif" width="306" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>3.1 Local Vs International sunscreens</strong></p>\r\n<p>\r\n	<strong>(i) SPF label accuracy</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Coefficient of adjustment (C) was used as a standard to measure the accuracy of SPF label of the sunscreen. It was calculated by using formula 4. Values between 0.8-1.2 meant the SPF label was accurate. Figure 2 shows SPF label and in vitro SPF of various sunscreens.</p>\r\n<p>\r\n	Formula 4: Calculation for Coefficient of Adjustment</p>\r\n<p>\r\n	&nbsp;<img height="43" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image010.gif" width="275" /></p>\r\n<p>\r\n	Where,</p>\r\n<p>\r\n	E (&lambda;)&nbsp;&nbsp;&nbsp; = Erythema Action Spectrum at wavelength &lambda;.</p>\r\n<p>\r\n	I (&lambda;) &nbsp;&nbsp;&nbsp; = Spectral Irradiance of UV source at wavelength &lambda;.</p>\r\n<p>\r\n	d &lambda;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp; = Wavelength step = 1nm</p>\r\n<p>\r\n	A (&lambda;)&nbsp;&nbsp;&nbsp;&nbsp; = Absorbance of the test samples at wavelength &lambda;.</p>\r\n<p>\r\n	C&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; = Coefficient of adjustment</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp;</p>\r\n<p align="center">\r\n	Figure 2: Comparison between Local and International Sunscreens in terms of SPF Label Accuracy</p>\r\n<p>\r\n	&nbsp;<img height="264" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image012.jpg" width="624" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; A total of nine (100%) international sample plates had accurate SPF label while only three (33.3%) local sample plates had C values that fell between 0.8-1.2. A Fisher&rsquo;s exact test was computed to test the association between manufacture places and the SPF label accuracy. The result revealed that there was significant association between the manufacture place and the SPF label accuracy. The international sunscreens had more accurate SPF label than local sunscreens (p=0.009). The details of the analysis are shown in table 4.</p>\r\n<p align="center">\r\n	Table 4: Analysis of the Comparison between Local and International Sunscreens in terms of SPF Label Accuracy</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:108px;height:26px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td colspan="3" style="width:246px;height:26px;">\r\n				<p>\r\n					SPF Accuracy</p>\r\n			</td>\r\n			<td rowspan="2" style="width:96px;height:26px;">\r\n				<p>\r\n					&nbsp;</p>\r\n				<p>\r\n					Total</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td rowspan="2" style="width:114px;height:26px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td colspan="2" rowspan="2" style="width:60px;height:26px;">\r\n				<p>\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:108px;height:26px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td colspan="2" style="width:115px;height:26px;">\r\n				<p>\r\n					Acceptable</p>\r\n			</td>\r\n			<td style="width:132px;height:26px;">\r\n				<p>\r\n					Not Acceptable</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:108px;height:18px;">\r\n				<p>\r\n					International</p>\r\n			</td>\r\n			<td style="width:114px;height:18px;">\r\n				<p>\r\n					9 (100.0%)</p>\r\n			</td>\r\n			<td colspan="2" style="width:132px;height:18px;">\r\n				<p>\r\n					0 (0.0%)</p>\r\n			</td>\r\n			<td style="width:96px;height:18px;">\r\n				<p>\r\n					9 (100.0%)</p>\r\n			</td>\r\n			<td rowspan="2" style="width:114px;height:18px;">\r\n				<p>\r\n					Fisher&#39;s Exact Test</p>\r\n			</td>\r\n			<td rowspan="2" style="width:60px;height:18px;">\r\n				<p>\r\n					0.009</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:108px;height:18px;">\r\n				<p>\r\n					Local</p>\r\n			</td>\r\n			<td style="width:114px;height:18px;">\r\n				<p>\r\n					3 (33.3%)</p>\r\n			</td>\r\n			<td colspan="2" style="width:132px;height:18px;">\r\n				<p>\r\n					6 (66.7%)</p>\r\n			</td>\r\n			<td style="width:96px;height:18px;">\r\n				<p>\r\n					9 (100.0%)</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:108px;height:18px;">\r\n				<p>\r\n					Total</p>\r\n			</td>\r\n			<td colspan="2" style="width:115px;height:18px;">\r\n				<p>\r\n					12</p>\r\n			</td>\r\n			<td style="width:132px;height:18px;">\r\n				<p>\r\n					6</p>\r\n			</td>\r\n			<td style="width:96px;height:18px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:114px;height:18px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td colspan="2" style="width:60px;height:18px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n		</tr>\r\n		<tr height="0">\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n			<td>\r\n				&nbsp;</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	&nbsp;(ii) Relative UVAPF level</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; According to European Commission 2006, sunscreens should provide UVAPF at least one third of the SPF label to ensure balanced and adequate protection against both UVA and UVB radiation. Figure 3 shows the comparison of relative UVAPF level between local and international sunscreens. There were only two (22.2%) international sunscreens with acceptable UVAPF to SPF label ratio. Fisher&rsquo;s exact test proved that most of the sunscreens regardless of the manufacture place were not providing enough UVA protection (p=0.471) as shown in table 5.</p>\r\n<p align="center">\r\n	&nbsp;</p>\r\n<p align="center">\r\n	Figure 3: Comparison of Relative UVAPF Level between Local and International Sunscreens</p>\r\n<p>\r\n	<img height="289" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image014.gif" width="624" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<br clear="ALL" />\r\n<p align="center">\r\n	Table 5: Analysis for the Comparison of Relative UVAPF Level between Local And International Sunscreens</p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td rowspan="2" style="width:102px;height:26px;">\r\n				<p style="margin-left:23.8pt;">\r\n					&nbsp;</p>\r\n				<p style="margin-left:23.8pt;">\r\n					&nbsp;</p>\r\n			</td>\r\n			<td colspan="2" style="width:246px;height:26px;">\r\n				<p style="margin-left:23.8pt;">\r\n					Relative UVAPF Level</p>\r\n			</td>\r\n			<td rowspan="2" style="width:90px;height:26px;">\r\n				<p style="margin-left:23.8pt;">\r\n					&nbsp;</p>\r\n				<p style="margin-left:-4.65pt;">\r\n					Total</p>\r\n				<p style="margin-left:23.8pt;">\r\n					&nbsp;</p>\r\n			</td>\r\n			<td rowspan="2" style="width:114px;height:26px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td rowspan="2" style="width:72px;height:26px;">\r\n				<p style="margin-left:-4.65pt;">\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:120px;height:26px;">\r\n				<p style="margin-left:-4.65pt;">\r\n					Acceptable</p>\r\n			</td>\r\n			<td style="width:126px;height:26px;">\r\n				<p style="margin-left:-.15pt;">\r\n					Not Acceptable</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:102px;height:18px;">\r\n				<p style="margin-left:-.15pt;">\r\n					International</p>\r\n			</td>\r\n			<td style="width:120px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					2(22.2%)</p>\r\n			</td>\r\n			<td style="width:126px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					7 (77.8%)</p>\r\n			</td>\r\n			<td style="width:90px;height:18px;">\r\n				<p>\r\n					9 (100.0%)</p>\r\n			</td>\r\n			<td rowspan="3" style="width:114px;height:18px;">\r\n				<p>\r\n					Fisher&#39;s Exact Test</p>\r\n			</td>\r\n			<td rowspan="3" style="width:72px;height:18px;">\r\n				<p>\r\n					0.471</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:102px;height:18px;">\r\n				<p style="margin-left:-.15pt;">\r\n					Local</p>\r\n			</td>\r\n			<td style="width:120px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					0 (0.0%)</p>\r\n			</td>\r\n			<td style="width:126px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					9 (100.0%)</p>\r\n			</td>\r\n			<td style="width:90px;height:18px;">\r\n				<p style="margin-left:-.15pt;">\r\n					9 (100.0%)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:102px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					Total</p>\r\n			</td>\r\n			<td style="width:120px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					2</p>\r\n			</td>\r\n			<td style="width:126px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					16</p>\r\n			</td>\r\n			<td style="width:90px;height:18px;">\r\n				<p style="margin-left:23.8pt;">\r\n					18</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	(iii) Photostability</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Figure 4 displays the photostability of international and local sunscreens. Local sunscreens scored a mean rank of 13.22 while the mean rank for international sunscreens was 5.78 only in Mann-Whitney U test (Table 6). Hence, local sunscreens were more photostable than international sunscreens (p=0.003).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p align="center">\r\n	Figure 4: Comparison of Photostability between Local and International Sunscreens</p>\r\n<p>\r\n	&nbsp;<img height="289" src="file:///C:/DOCUME~1/enter/LOCALS~1/Temp/msohtmlclip1/01/clip_image016.gif" width="624" /></p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p align="center">\r\n	Table 6: Analysis for the Comparison of Photostability between Local and International Sunscreens</p>\r\n<table border="1" cellpadding="0" cellspacing="0" style="width:631px;" width="631">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:139px;height:66px;">\r\n				<p>\r\n					Manufacture Place</p>\r\n			</td>\r\n			<td style="width:34px;height:66px;">\r\n				<p>\r\n					N</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:70px;height:66px;">\r\n				<p>\r\n					Median</p>\r\n			</td>\r\n			<td style="width:104px;height:66px;">\r\n				<p>\r\n					Interquatile range</p>\r\n			</td>\r\n			<td style="width:104px;height:66px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td style="width:96px;height:66px;">\r\n				<p>\r\n					Mean Rank</p>\r\n			</td>\r\n			<td style="width:84px;height:66px;">\r\n				<p>\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:139px;height:15px;">\r\n				<p>\r\n					International</p>\r\n			</td>\r\n			<td style="width:34px;height:15px;">\r\n				<p>\r\n					9</p>\r\n			</td>\r\n			<td style="width:70px;height:15px;">\r\n				<p>\r\n					30.13</p>\r\n			</td>\r\n			<td style="width:104px;height:15px;">\r\n				<p>\r\n					6.31</p>\r\n			</td>\r\n			<td rowspan="3" style="width:104px;height:15px;">\r\n				<p>\r\n					Mann-Whitney U</p>\r\n			</td>\r\n			<td style="width:96px;height:15px;">\r\n				<p>\r\n					5.78</p>\r\n			</td>\r\n			<td rowspan="3" style="width:84px;height:15px;">\r\n				<p>\r\n					p =0.003</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:139px;height:23px;">\r\n				<p>\r\n					Local</p>\r\n			</td>\r\n			<td style="width:34px;height:23px;">\r\n				<p>\r\n					9</p>\r\n			</td>\r\n			<td style="width:70px;height:23px;">\r\n				<p>\r\n					55.4</p>\r\n			</td>\r\n			<td style="width:104px;height:23px;">\r\n				<p>\r\n					35.72</p>\r\n			</td>\r\n			<td style="width:96px;height:23px;">\r\n				<p>\r\n					13.22</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:139px;height:15px;">\r\n				<p>\r\n					Total</p>\r\n			</td>\r\n			<td style="width:34px;height:15px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:70px;height:15px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:104px;height:15px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:96px;height:15px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>3.2 Roles of Antioxidant Creams in Photoprotection</strong></p>\r\n<p>\r\n	(i)&nbsp; Differences Before and After Adding Antioxidants onto Sunscreens</p>\r\n<p>\r\n	A comparison between in vitro SPF, in vitro UVAPF and photostability of sunscreens alone, sunscreens added with Vitamin C cream, sunscreens added with Vitamin E cream and sunscreens added with a combination of Vitamins C and E cream was performed. Table 7 shows the differences before and after adding antioxidants into sunscreens according to the above mentioned combination. With reference to the Kruskal-Wallis tests, addition of&nbsp; either Vitamin C or E creams with sunscreens improved the in vitro SPF (p=0.009), in vitro UVAPF (p=0.007), and photostability (p=0.002).</p>\r\n<p align="center">\r\n	Table 7: Differences Before and After Adding Antioxidants onto Sunscreens</p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:103px;height:41px;">\r\n				<p>\r\n					Parameter</p>\r\n			</td>\r\n			<td style="width:117px;height:41px;">\r\n				<p>\r\n					Combination</p>\r\n			</td>\r\n			<td style="width:32px;height:41px;">\r\n				<p>\r\n					N</p>\r\n			</td>\r\n			<td style="width:68px;height:41px;">\r\n				<p>\r\n					Median</p>\r\n			</td>\r\n			<td style="width:97px;height:41px;">\r\n				<p>\r\n					Interquatile Range</p>\r\n			</td>\r\n			<td style="width:69px;height:41px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td style="width:85px;height:41px;">\r\n				<p>\r\n					Mean Rank</p>\r\n			</td>\r\n			<td style="width:60px;height:41px;">\r\n				<p>\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="4" style="width:103px;height:17px;">\r\n				<p>\r\n					In vitro SPF</p>\r\n			</td>\r\n			<td style="width:117px;height:17px;">\r\n				<p>\r\n					Sunscreen only</p>\r\n			</td>\r\n			<td style="width:32px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:17px;">\r\n				<p>\r\n					33.56</p>\r\n			</td>\r\n			<td style="width:97px;height:17px;">\r\n				<p>\r\n					71.38</p>\r\n			</td>\r\n			<td rowspan="4" style="width:69px;height:17px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:85px;height:17px;">\r\n				<p>\r\n					25.17</p>\r\n			</td>\r\n			<td rowspan="4" style="width:60px;height:17px;">\r\n				<p>\r\n					p =0.009</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:34px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:32px;height:34px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:34px;">\r\n				<p>\r\n					115.9</p>\r\n			</td>\r\n			<td style="width:97px;height:34px;">\r\n				<p>\r\n					287</p>\r\n			</td>\r\n			<td style="width:85px;height:34px;">\r\n				<p>\r\n					45.67</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:38px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:32px;height:38px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:38px;">\r\n				<p>\r\n					77.82</p>\r\n			</td>\r\n			<td style="width:97px;height:38px;">\r\n				<p>\r\n					199.5</p>\r\n			</td>\r\n			<td style="width:85px;height:38px;">\r\n				<p>\r\n					31.72</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:60px;">\r\n				<p>\r\n					Sunscreen + Vitamin C and E</p>\r\n			</td>\r\n			<td style="width:32px;height:60px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:60px;">\r\n				<p>\r\n					67.77</p>\r\n			</td>\r\n			<td style="width:97px;height:60px;">\r\n				<p>\r\n					239.2</p>\r\n			</td>\r\n			<td style="width:85px;height:60px;">\r\n				<p>\r\n					43.44</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="4" style="width:103px;height:17px;">\r\n				<p>\r\n					In vitro UVAPF</p>\r\n			</td>\r\n			<td style="width:117px;height:17px;">\r\n				<p>\r\n					Sunscreen only</p>\r\n			</td>\r\n			<td style="width:32px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:17px;">\r\n				<p>\r\n					5.35</p>\r\n			</td>\r\n			<td style="width:97px;height:17px;">\r\n				<p>\r\n					5.13</p>\r\n			</td>\r\n			<td rowspan="4" style="width:69px;height:17px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:85px;height:17px;">\r\n				<p>\r\n					25.72</p>\r\n			</td>\r\n			<td rowspan="4" style="width:60px;height:17px;">\r\n				<p>\r\n					p =0.007</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:40px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:32px;height:40px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:40px;">\r\n				<p>\r\n					10.63</p>\r\n			</td>\r\n			<td style="width:97px;height:40px;">\r\n				<p>\r\n					15.41</p>\r\n			</td>\r\n			<td style="width:85px;height:40px;">\r\n				<p>\r\n					45.78</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:35px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:32px;height:35px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:35px;">\r\n				<p>\r\n					5.98</p>\r\n			</td>\r\n			<td style="width:97px;height:35px;">\r\n				<p>\r\n					12.06</p>\r\n			</td>\r\n			<td style="width:85px;height:35px;">\r\n				<p>\r\n					30.44</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:58px;">\r\n				<p>\r\n					Sunscreen + Vitamin C and E</p>\r\n			</td>\r\n			<td style="width:32px;height:58px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:58px;">\r\n				<p>\r\n					9.63</p>\r\n			</td>\r\n			<td style="width:97px;height:58px;">\r\n				<p>\r\n					12.26</p>\r\n			</td>\r\n			<td style="width:85px;height:58px;">\r\n				<p>\r\n					44.06</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="4" style="width:103px;height:17px;">\r\n				<p>\r\n					Photostability</p>\r\n			</td>\r\n			<td style="width:117px;height:17px;">\r\n				<p>\r\n					Sunscreen only</p>\r\n			</td>\r\n			<td style="width:32px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:17px;">\r\n				<p>\r\n					33.22</p>\r\n			</td>\r\n			<td style="width:97px;height:17px;">\r\n				<p>\r\n					26.9</p>\r\n			</td>\r\n			<td rowspan="4" style="width:69px;height:17px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:85px;height:17px;">\r\n				<p>\r\n					36.5</p>\r\n			</td>\r\n			<td rowspan="4" style="width:60px;height:17px;">\r\n				<p>\r\n					p =0.002</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:34px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:32px;height:34px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:34px;">\r\n				<p>\r\n					22.46</p>\r\n			</td>\r\n			<td style="width:97px;height:34px;">\r\n				<p>\r\n					12.51</p>\r\n			</td>\r\n			<td style="width:85px;height:34px;">\r\n				<p>\r\n					21.22</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:39px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:32px;height:39px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:39px;">\r\n				<p>\r\n					54.66</p>\r\n			</td>\r\n			<td style="width:97px;height:39px;">\r\n				<p>\r\n					29.8</p>\r\n			</td>\r\n			<td style="width:85px;height:39px;">\r\n				<p>\r\n					47.06</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:117px;height:3px;">\r\n				<p>\r\n					Sunscreen + Vitamin C and E</p>\r\n			</td>\r\n			<td style="width:32px;height:3px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:68px;height:3px;">\r\n				<p>\r\n					60.04</p>\r\n			</td>\r\n			<td style="width:97px;height:3px;">\r\n				<p>\r\n					66.44</p>\r\n			</td>\r\n			<td style="width:85px;height:3px;">\r\n				<p>\r\n					41.22</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	(ii) Differences between the Photoprotection Levels of Vitamins C and E cream</p>\r\n<p>\r\n	Vitamin C cream enhanced the SPF of the sunscreens (p= 0.040) while Vitamin E made the sunscreens more photostable (p=0.000). However, there is no significant differences between Vitamin C and E in improving UVAPF (p=0.054) of the sunscreens (Table 8).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p align="center">\r\n	Table 8: Effects of Adding Vitamin C or Vitamin E Creams Onto Sunscreens</p>\r\n<table border="1" cellpadding="0" cellspacing="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:107px;height:47px;">\r\n				<p>\r\n					Parameter</p>\r\n			</td>\r\n			<td style="width:98px;height:47px;">\r\n				<p>\r\n					Combination</p>\r\n			</td>\r\n			<td style="width:36px;height:47px;">\r\n				<p>\r\n					N</p>\r\n			</td>\r\n			<td style="width:72px;height:47px;">\r\n				<p>\r\n					Median</p>\r\n			</td>\r\n			<td style="width:90px;height:47px;">\r\n				<p>\r\n					Interquatile range</p>\r\n			</td>\r\n			<td style="width:96px;height:47px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td style="width:72px;height:47px;">\r\n				<p>\r\n					Mean Rank</p>\r\n			</td>\r\n			<td style="width:60px;height:47px;">\r\n				<p>\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="2" style="width:107px;height:35px;">\r\n				<p>\r\n					In vitro SPF</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:98px;height:35px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:35px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:35px;">\r\n				<p>\r\n					115.9</p>\r\n			</td>\r\n			<td style="width:90px;height:35px;">\r\n				<p>\r\n					287</p>\r\n			</td>\r\n			<td rowspan="2" style="width:96px;height:35px;">\r\n				<p>\r\n					Mann-Whitney U = 97.00</p>\r\n			</td>\r\n			<td style="width:72px;height:35px;">\r\n				<p>\r\n					22.11</p>\r\n			</td>\r\n			<td rowspan="2" style="width:60px;height:35px;">\r\n				<p>\r\n					p =0.040</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:98px;height:40px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:40px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:40px;">\r\n				<p>\r\n					77.82</p>\r\n			</td>\r\n			<td style="width:90px;height:40px;">\r\n				<p>\r\n					199.5</p>\r\n			</td>\r\n			<td style="width:72px;height:40px;">\r\n				<p>\r\n					14.89</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="2" style="width:107px;height:35px;">\r\n				<p>\r\n					In vitro UVAPF</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:98px;height:35px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:35px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:35px;">\r\n				<p>\r\n					10.63</p>\r\n			</td>\r\n			<td style="width:90px;height:35px;">\r\n				<p>\r\n					15.41</p>\r\n			</td>\r\n			<td rowspan="2" style="width:96px;height:35px;">\r\n				<p>\r\n					Mann-Whitney U =101.00</p>\r\n			</td>\r\n			<td style="width:72px;height:35px;">\r\n				<p>\r\n					21.89</p>\r\n			</td>\r\n			<td rowspan="2" style="width:60px;height:35px;">\r\n				<p>\r\n					p =0.054</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:98px;height:34px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:34px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:34px;">\r\n				<p>\r\n					5.98</p>\r\n			</td>\r\n			<td style="width:90px;height:34px;">\r\n				<p>\r\n					12.06</p>\r\n			</td>\r\n			<td style="width:72px;height:34px;">\r\n				<p>\r\n					15.11</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:107px;height:38px;">\r\n				<p>\r\n					Photostability</p>\r\n			</td>\r\n			<td style="width:98px;height:38px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:38px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:38px;">\r\n				<p>\r\n					22.46</p>\r\n			</td>\r\n			<td style="width:90px;height:38px;">\r\n				<p>\r\n					12.51</p>\r\n			</td>\r\n			<td rowspan="2" style="width:96px;height:38px;">\r\n				<p>\r\n					Mann-Whitney U =38.00</p>\r\n			</td>\r\n			<td style="width:72px;height:38px;">\r\n				<p>\r\n					11.61</p>\r\n			</td>\r\n			<td rowspan="2" style="width:60px;height:38px;">\r\n				<p>\r\n					p =0.000</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:107px;height:41px;">\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n			<td style="width:98px;height:41px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:41px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:72px;height:41px;">\r\n				<p>\r\n					54.66</p>\r\n			</td>\r\n			<td style="width:90px;height:41px;">\r\n				<p>\r\n					29.8</p>\r\n			</td>\r\n			<td style="width:72px;height:41px;">\r\n				<p>\r\n					25.39</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	(iii) The Synergistic Effects of combining both Vitamins C and E cream.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; We observed that combining both Vitamins C and E cream into a sunscreen did not significantly improve the SPF and UVAPF (p&gt;0.005) compared to the use of a single antioxidant cream. More importantly, the above combination also showed lower photostability (p=0.002) compared to adding Vitamin E alone (Table 9). .</p>\r\n<p align="center">\r\n	Table 9: Synergistic Effects of Combining Both Vitamins C And E Cream</p>\r\n<table border="1" cellpadding="0" cellspacing="0" width="666">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:108px;height:23px;">\r\n				<p>\r\n					Parameter</p>\r\n			</td>\r\n			<td style="width:180px;height:23px;">\r\n				<p>\r\n					Combination</p>\r\n			</td>\r\n			<td style="width:36px;height:23px;">\r\n				<p>\r\n					N</p>\r\n			</td>\r\n			<td style="width:66px;height:23px;">\r\n				<p>\r\n					Median</p>\r\n			</td>\r\n			<td style="width:96px;height:23px;">\r\n				<p>\r\n					Interquatile Range</p>\r\n			</td>\r\n			<td style="width:66px;height:23px;">\r\n				<p>\r\n					Test</p>\r\n			</td>\r\n			<td style="width:60px;height:23px;">\r\n				<p>\r\n					Mean rank</p>\r\n			</td>\r\n			<td style="width:54px;height:23px;">\r\n				<p>\r\n					p</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="3" style="width:108px;height:28px;">\r\n				<p>\r\n					In vitro SPF</p>\r\n			</td>\r\n			<td style="width:180px;height:28px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:28px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:28px;">\r\n				<p>\r\n					115.9</p>\r\n			</td>\r\n			<td style="width:96px;height:28px;">\r\n				<p>\r\n					287</p>\r\n			</td>\r\n			<td rowspan="3" style="width:66px;height:28px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:60px;height:28px;">\r\n				<p>\r\n					31.39</p>\r\n			</td>\r\n			<td rowspan="3" style="width:54px;height:28px;">\r\n				<p>\r\n					p =0.100</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:32px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:32px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:32px;">\r\n				<p>\r\n					77.82</p>\r\n			</td>\r\n			<td style="width:96px;height:32px;">\r\n				<p>\r\n					199.5</p>\r\n			</td>\r\n			<td style="width:60px;height:32px;">\r\n				<p>\r\n					21.06</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:43px;">\r\n				<p>\r\n					Sunscreen + Vitamin C + E</p>\r\n			</td>\r\n			<td style="width:36px;height:43px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:43px;">\r\n				<p>\r\n					67.77</p>\r\n			</td>\r\n			<td style="width:96px;height:43px;">\r\n				<p>\r\n					239.2</p>\r\n			</td>\r\n			<td style="width:60px;height:43px;">\r\n				<p>\r\n					30.06</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="3" style="width:108px;height:35px;">\r\n				<p>\r\n					In vitro UVAPF</p>\r\n			</td>\r\n			<td style="width:180px;height:35px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:35px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:35px;">\r\n				<p>\r\n					10.63</p>\r\n			</td>\r\n			<td style="width:96px;height:35px;">\r\n				<p>\r\n					15.41</p>\r\n			</td>\r\n			<td rowspan="3" style="width:66px;height:35px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:60px;height:35px;">\r\n				<p>\r\n					31.44</p>\r\n			</td>\r\n			<td rowspan="3" style="width:54px;height:35px;">\r\n				<p>\r\n					p =0.115</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:34px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:34px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:34px;">\r\n				<p>\r\n					5.98</p>\r\n			</td>\r\n			<td style="width:96px;height:34px;">\r\n				<p>\r\n					12.06</p>\r\n			</td>\r\n			<td style="width:60px;height:34px;">\r\n				<p>\r\n					21.28</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:41px;">\r\n				<p>\r\n					Sunscreen + Vitamin C + E</p>\r\n			</td>\r\n			<td style="width:36px;height:41px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:41px;">\r\n				<p>\r\n					9.63</p>\r\n			</td>\r\n			<td style="width:96px;height:41px;">\r\n				<p>\r\n					12.26</p>\r\n			</td>\r\n			<td style="width:60px;height:41px;">\r\n				<p>\r\n					29.78</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td rowspan="3" style="width:108px;height:29px;">\r\n				<p>\r\n					Photostability</p>\r\n			</td>\r\n			<td style="width:180px;height:29px;">\r\n				<p>\r\n					Sunscreen + Vitamin C</p>\r\n			</td>\r\n			<td style="width:36px;height:29px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:29px;">\r\n				<p>\r\n					22.46</p>\r\n			</td>\r\n			<td style="width:96px;height:29px;">\r\n				<p>\r\n					12.51</p>\r\n			</td>\r\n			<td rowspan="3" style="width:66px;height:29px;">\r\n				<p>\r\n					Kruskal Wallis</p>\r\n			</td>\r\n			<td style="width:60px;height:29px;">\r\n				<p>\r\n					17.22</p>\r\n			</td>\r\n			<td rowspan="3" style="width:54px;height:29px;">\r\n				<p>\r\n					p =0.02</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:17px;">\r\n				<p>\r\n					Sunscreen + Vitamin E</p>\r\n			</td>\r\n			<td style="width:36px;height:17px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:17px;">\r\n				<p>\r\n					54.66</p>\r\n			</td>\r\n			<td style="width:96px;height:17px;">\r\n				<p>\r\n					29.8</p>\r\n			</td>\r\n			<td style="width:60px;height:17px;">\r\n				<p>\r\n					34.5</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td style="width:180px;height:34px;">\r\n				<p>\r\n					Sunscreen + Vitamin C + E</p>\r\n			</td>\r\n			<td style="width:36px;height:34px;">\r\n				<p>\r\n					18</p>\r\n			</td>\r\n			<td style="width:66px;height:34px;">\r\n				<p>\r\n					60.04</p>\r\n			</td>\r\n			<td style="width:96px;height:34px;">\r\n				<p>\r\n					66.44</p>\r\n			</td>\r\n			<td style="width:60px;height:34px;">\r\n				<p>\r\n					30.78</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>4. DISCUSSIONS</strong></p>\r\n<p>\r\n	<strong>4.1 Local Vs International sunscreens</strong></p>\r\n<p>\r\n	(i) SPF label accuracy</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Results of analysis showed that local sunscreens had less accurate SPF label than international sunscreens probably because the selected locally produced sunscreens did not comply with the Good Manufacturing Practice (GMP) guidelines which recommend products to be tested in terms of quality, packaging, safety, toxicology and labeling before being awarded with the certificate.&nbsp;&nbsp;</p>\r\n<p>\r\n	(ii) Relative UVAPF level</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Our result is consistent with the finding from Environmental Work Group (EWG) 6th Annual Sunscreen Guide 2012 where most of the sunscreens were found to provide inadequate UVA protection. However information regarding UVAPF provided by the sunscreens was not a requirement in passing the Safety Assessment of Cosmetic Product in any countries. Most sunscreens were found to screen out UVB only. Thus, prolonged exposure to sunlight does increase the risk of melanoma despite the use of sunscreens (Gorham, 2007).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	(iii) Photostability</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; After two hours of irradiation under the sun, all sunscreen samples had decreased in vitro SPF values. This might be due to degradation of UV filters with exposure to irradiation which decreases the photostability of sunscreens thus leading to lower SPF values.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; According to the EWG Skin Deep Sunscreen Guideline 2011, low‐SPF products contained fewer amounts of UV filters than high‐SPF sunscreens. Consumers are not recommended to use sunscreens with high SPF values because degradation of excessive UV filters during exposure to sunlight produces ROS which damages the skin (XX). Sunscreens with high SPF values are thus suitable only for short duration of sun exposure. In our study, SPF values for locally produced sunscreens were 50% lower than the international sunscreens. Hence, locally produced sunscreens are suggested to be more photostable than international sunscreens.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;</p>\r\n<p>\r\n	<strong>4.2 Roles of Antioxidant Creams in Photoprotection</strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Using vitamin C, vitamin E or the combination of both creams together with sunscreens are found to improve the function of sunscreens in terms of SPF, UVAPF and photostability.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; According to Dominique Lutz et al. (1999), SPF and UVAPF values are affected by the concentration of UV filters and the thickness of sunscreens applied. This is in line with our study which found that the increased thickness of applied sunscreens upon application of antioxidant creams showed increased values of SPF and UVAPF. In other words, antioxidant creams&nbsp; are eable to enhance the photoprotection of sunscreens without increasing the concentration of UV filters.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; In addition, photostability of the sunscreens is also improved with the use of antioxidant creams. This may be due to the capability of antioxidants to neutralize free radicals generated from UV light and degraded UV filters (Edlich et al. 2004). This in turn prevents subsequent generation of ROS which disrupts the integrity of sunscreen leading to increased photostability.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Vitamin C cream increased the in vitro SPF level more than vitamin E cream.&nbsp; Both Since vitamins C and E has been shown to have no UVB filtering functions (John C. Murray, 2008), The increase in SPF value could have been contributed by minor additives in the Vitamin C cream such as Ethylhexyl Methoxycinnamate and Butyl Methoxydibenzolmethane which are UVB filters.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; For UVAPF, there is no significant difference between vitamins C and E in improving UVAPF of the sunscreens. As mention before, antioxidant cream act as supplement to the sunscreen since they have no UV filtering properties. They improved UVAPF and SPF value solely via the dose dependant effect. This explained our observation as the thickness applied for both vitamin C and E onto the sunscreen is the same.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Meanwhile, vitamin E cream showed higher efficacy in improving the photostability of the sunscreen compared to vitamin C. &alpha;-Tocopherol acetate (Vitamin E) is stable and will not be converted to its unstable free form &alpha;-tocopherol if applied topically (Alberts et al, 1996). On the other hand, L- ascorbic acid (Vitamin C) will always be metabolized to its unstable counterpart by the skin (Sheldon R.P et al, 2001).&nbsp; Though effort had been put in esterifying L-ascobic acid to make it more stable in topical cream, unfortunately it is still less stable than &alpha;-tocopherol.</p>\r\n<p>\r\n	(iii) The Synergistic Effects of combining both Vitamins C and E cream.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The synergistic effects of combining both vitamins C and E cream were not observed in our experiment. This combination improved the SPF and UVAPF values insignificantly compared to adding Vitamin C or E alone. This is contradictory to findings of other researches. The explanation for this insignificance is probably due to the same thickness that we applied for the three combinations.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Combining both Vitamin C and E onto sunscreens demonstrated lower photostability in comparison to adding Vitamin E alone. We postulated that portion of degraded Vitamin C reduces the stability of this combination.</p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; &nbsp;</p>\r\n<p>\r\n	<strong>CONSCLUSION </strong></p>\r\n<p>\r\n	&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; The use of sunscreen alone is not safe and effective regardless the manufacture place. Consumers are advised to use the sunscreen with Vitamin C for intense sun exposure while Vitamin E for longer uncovering.</p>\r\n<p>\r\n	<strong>ACKNOWLEDGEMENT</strong></p>\r\n<p>\r\n	&bull;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Department of Biochemistry and Physiology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Aziz, 50300 Kuala Lumpur, Malaysia</p>\r\n<p>\r\n	&bull;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Malaysian Meteorological Department, Petaling Jaya, Selangor, Malaysia</p>\r\n<p>\r\n	&bull;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; This research is approved by ethical committee and funded by Dana Fundamental UKM (320007001)</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>REFERENCES</strong></p>\r\n<p style="margin-left:36.0pt;">\r\n	Alberts DS. Goldman R. Xu MJ. Dorr RT. Quinn J. Welch K. Guillen-Rodriguez J. Aickin M. Peng YM. Loescher L. Gensler H., 1996. Disposition and metabolism of topically administered alpha-tocopherol acetate: a common ingredient of commercially available sunscreens and cosmetics. Journal of Nutrition &amp; Cancer. 26(2):193-201, 1996.</p>\r\n<p style="margin-left:36.0pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:36.0pt;">\r\n	Association of Southeast Asian Nations (ASEAN) Safety Assessment of Cosmetic Product, &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Guideline for Control of Cosmetic Products in Malaysia, National Pharmaceutical &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Control Bureau.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Available at http://portal.bpfk.gov.my/index.cfm?menuid=63&amp;parentid=34</p>\r\n<p style="margin-left:36.0pt;">\r\n	Bastuji-Garin, S., Diepgen, T. L. 2002.&nbsp; Cutaneous malignant melanoma, sun exposure and sunscreen use: epidemiological evidence. Journal of Dermatology. 146:24&ndash;30.</p>\r\n<p style="margin-left:36.0pt;">\r\n	COLIPA Guidelines: In vitro Method for the Determination of the UVA protection factor and Critical Wavelength Values of Sunscreen Product.COLIPA, p.24.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Diffey, B.L, Tanner,P.R, Matts P.J, Nash J.F, 2000. In vitro assessment of the broad spectrum</p>\r\n<p style="margin-left:36.0pt;">\r\n	ultraviolet protection of sunscreen producs. Journal of America Accademy of Dermatology.43, 1024-1035.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Dominique L., UV Sun conference filters Paris Nov 99, Contribution to measuring In Vitro</p>\r\n<p style="margin-left:36.0pt;">\r\n	protection: Some key rules for the reliability of sunscreen spectrophotometric evaluation. Available at http://www.biblioscreen.helioscreen.fr/Documents%20helioscreen/Conference_UV_SUN_%20FILTER_%20Nov99.pdf</p>\r\n<p style="margin-left:36.0pt;">\r\n	EC, 2006. European Comission Recommendation on efficacy of sunscreen product and the claims of relating thereto. 2006/647/EC.OJ L 265, 26.9.2006, 39-43.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Environmental Work Group (EWG) 6th Annual Sunscreen Guide 2012. Available at http://breakingnews.ewg.org/2012sunscreen/</p>\r\n<p style="margin-left:36.0pt;">\r\n	Edlich, R.F., Winters, K.L., Lim, H.W., Cox, M.J., Becker, D.G., Horowitz, J.H., Nitcher, L.S., Britt, L.D., Long, W.B. 2004.&nbsp; Photoprotection by Sunscreens with Topical Antioxidants and Systemic Antioxidants to Reduce Sun Exposure. Journal of Long-Term Effect of Medical Implants 14(4)317&ndash;340.</p>\r\n<p style="margin-left:36.0pt;">\r\n	FDA, 2011. Food and Drug Administration. FDA announces new requirements for over-the-counter (OTC) sunscreen products marketed in the U.S. Available at http://www.fda.gov/Drugs/ResourcesForYou/Consumers/BuyingUsingMedicineSafely/UnderstandingOver-the-CounterMedicines/ucm258468.htm</p>\r\n<p style="margin-left:36.0pt;">\r\n	Garoli, D., Pelizzo, M.G., Bernardini, B., Nicolosi, P., Alaibac, M. 2008. Sunscreen tests: correspondence between in vitrodata and values reported by the manufacturers. Journal of Dermatological Sciences. 52(3):193-204.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Gilchrest, B.A. 2008. Sun exposure and vitamin D sufficiency. American Journal of Clinical Nutrition 88(2): 570S-577S</p>\r\n<p style="margin-left:36.0pt;">\r\n	Gorham ED, Mohr SB, Garland CF, Chaplin G, Garland FC, 2007. Do suscreen increase risk of melanoma in populations residing at higher latitudes? Annals of Epidemiology. 17(12) :956-63</p>\r\n<p style="margin-left:36.0pt;">\r\n	Herzog, B., Wehrle, M., Quass, K., 2009. Photostability of&nbsp; UV absorber systems in sunscreens.Journal of Photochemical Photobiology 85 (4) : 869-878</p>\r\n<p style="margin-left:36.0pt;">\r\n	John C. Murray, MD, James A. Burch, Robert D. Streilein, Mary Ann Iannacchione, Russell P. Hall, MD, and Sheldon R. Pinnell, MD, 2008.A topical antioxidant solution containing vitamins C and E stabilized by ferulic acid provides protection for human skin against damage caused by ultraviolet irradiation. Journal of American Academy of Dermatology .59 (3): 418-425</p>\r\n<p style="margin-left:36.0pt;">\r\n	Seite, S., Colige, A., Piquemal-Vivenot, P., et al. 2000.&nbsp; A full-UV spectrum absorbing daily use cream protects human skin against biological changes occurring in photoaging. Journal of Photodermatol Photoimmunol Photomed 16:147-55.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Steven, Q.Wang., Uli Osterwalder, and Katinka Jung. 2010. Ex vivo evaluation of radical sun protection factor in popular sunscreens with antioxidants. The American Academy of Dermatology.</p>\r\n<p style="margin-left:36.0pt;">\r\n	Sheldon, R.P, Huanshu Y, Mostafa O, Nancy M.R, Holly, V.DB., Linda, C.W.,Yaohui W., Mark L., 2001. Topical L-ascorbic acid : Percutaneous absorption studies. Dermatologic Surgery 27: 137-142.</p>\r\n', 1);
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(976, 971, '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Title: Short running title: Haemodialysis Affects Corneal Thickness but Not Intraocular Pressure Measurements </strong></p>\r\n<p>\r\n	<strong>ABSTRACT</strong></p>\r\n<p>\r\n	<strong>Objective</strong></p>\r\n<p>\r\n	To<em>study the effect of HD on CCT and IOP in patients undergoing HD </em></p>\r\n<p>\r\n	<strong>Method </strong></p>\r\n<p>\r\n	A hospital-based, prospective observational study was conducted <em>at the Haemodialysis Centre and Ophthalmology Clinic of the UKM Medical Centre (UKMMC), Kuala Lumpur, Malaysia. Fifty </em>eyes of 50 patients undergoing haemodialysis who fulfil the criteria were enrolled in the study using universal sampling method. Ocular examinationwas performed before haemodialysis. Pre- and post-haemodialysis IOP and CCT measurements of between 30 to 60 minutes before and after HD and total body volume measurements were performed for each patient. The recorded IOP measurements were then adjusted for CCT using this formula:Adjusted&nbsp; IOP (aIOP) = measured&nbsp; IOP -(CCT-454)/50 &times; 2.5 mmHg. Out of 50 eyes in total, 45 of right eyes were used for statistical analysis. In 5 cases, the right eyes did not meet the inclusion criteria so the left eyes were used for measurements The data were analysed using the SPSS version 19.0. Paired t-test was used to compare the IOP, CCT and body weight measurements before and after HD. Pearson correlation was used to investigate possible correlation between changes in IOP, CCT and total body volume after HD. A level of p &lt; 0.05 was considered as statistically significant.</p>\r\n<p>\r\n	<strong>Results</strong></p>\r\n<p>\r\n	There were no statistically significant differences in IOP before and after.(p = 0.798). Mean CCT decreased significantly after HD, from 537.0 &plusmn; 38.9 &micro;m to 532.5 &plusmn; 36.2 &micro;m (p = 0.004). No correlation was found between change in CCT and total body volume loss (Pearson correlation test, r = 0.237, p = 0.098), as well as between the change in CCT and change in IOP (p = 0.724).</p>\r\n<p>\r\n	<strong>Conclusion</strong></p>\r\n<p>\r\n	HD causes significant changes in CCT which should be accounted for when measuring the IOP at least 60 minutes after HD, especially in patients being followed for the diagnosis of glaucoma.However, HD does not result in IOP change.</p>\r\n<p>\r\n	<strong>Introduction</strong></p>\r\n<table border="0" cellpadding="0">\r\n	<tbody>\r\n		<tr>\r\n			<td style="width:604px;height:866px;">\r\n				<p>\r\n					Haemodialysis (HD) has become an increasinglyimportant treatment of patients with end stage renal failure (ESRF). In Malaysia, the number of dialysis patients has tripled in 10 years from 6689 in 2000 to more than 20,000 in 2009 to give a prevalence rate of at least 747/ 1,000,000 population in 2009<sup>1</sup>. Dialyzed patients are often ill and exhibit metabolic derangement. Thus, it becomes increasingly important to understand the physiological changes occurring during HDas a result of the rapid shifts in body fluid and solute content.</p>\r\n				<p>\r\n					A number of studies have investigated the systemic side effects and ocular complications of the HD process. Among the ocular complications associated with chronic haemodialysis are reduced corneal endothelium cell density<sup>1</sup><sup>2</sup>, ischaemic optic neuropathy<sup>3</sup>, and endogenous endophthalmitis<strong><sup>4</sup></strong>. Studies of the changes of intraocular pressure (IOP) during or after haemodialysis are conflicting with some studies finding an&nbsp; increase in IOP<sup>5-9</sup>,some no significant change<sup>10-12</sup>, and some studies indicating a decrease.<sup>12 -14</sup> In the case ofIOP increase, it is postulated that there is movement of water from the plasma into the aqueous humour due to an osmotic disequilibrium between the two compartments caused by a rapid decrease in plasma osmolarity.<sup>14</sup><sup>-18 </sup>A meta-analysis by Levy et al&nbsp; in 2005 concluded that the influence of HD on IOP was still unclear.<sup>19</sup>D results in the loss of a significant amount of total body volume in ESRF patients. The off-loading of fluid during HD may lead to corneal deturgescence and changes in CCT hence affecting IOP readings <sup>10,20</sup>. There is to date no conclusive study correlating the changes of IOP with CCT following HD. Our study objective was to determine the changes in CCT and IOP among ESRF patients undergoingHD at our centre. The study also examined the correlation between changes in total body volume and changes in CCT and/or IOP.</p>\r\n				<p>\r\n					<strong>M</strong><strong>ethods</strong></p>\r\n				<p>\r\n					This is a hospital-based, observational study<em>performed at the H</em>aemodialysis<em>Centre and Ophthalmology Clinic of the UKM Medical Centre (UKMMC), Kuala Lumpur, Malaysia. </em>Ethical approval for this study was first obtained from the Research and Ethics Committee, Faculty of Medicine, Universiti Kebangsaan Malaysia. <em>The inclusion criteria included a</em>ll patients with ESRF undergoing HD from June 2010 to 30 November 2010. Universal sampling method was adopted. All subjects had undergone 4 hours of HD sessions 3 day a week, by high performance dialyzers at a blood flow rate of 250 ml/min.&nbsp;</p>\r\n				<p>\r\n					Exclusion criteria was ocular surface opacity, scarring or oedema, contact lens usage in the preceding 3 days, patients on steroid therapy of any form, glaucoma patients or patients on topical anti-glaucoma eye drops, previous corneal injury or surgery including cataract and glaucoma surgery, history of ocular trauma, ill or uncooperative patients. The objective and details of the study were explained to all patients while written informed consent was obtained.</p>\r\n				<p>\r\n					Pre-HD evaluation comprised an interview about previous health problems including past history of ocular disease, ocular surgeries, ocular treatments including laser and topical medications. Demographic data and duration of HD was noted. Ophthalmic examination including IOP measurement with the Goldman aplanation tonometer, anterior segment examination and CCT measurement with the specular pachymetry (SP-200P, Topcon Corporation, Tokyo, Japan) which was performed for each patient 30-60 minutes before one single HD session. The same observer conducted each ophthalmic examination and was not masked to IOP or CCT readings.General examination included body weight measurement. CCT, IOP and body weight measurements were repeated 30-60 minutes after HD.</p>\r\n				<p>\r\n					CCT measurements were made at the centre of the cornea of the selected eye and the final measurement was based on an average of 3 readings (range &plusmn; 5 &micro;m). IOP measurement was done with the subjects in a sit-up position. Two acceptable readings within a range of &plusmn; 2mmHg were taken and the final measurement was the average of these two.</p>\r\n				<p>\r\n					A published nomogram for converting IOP based on CCT were used to determine the true IOP of each eye&nbsp; <sup>20</sup>. The correction factors were simplified for this study by using a baseline CCT of 545 mm, which was approximated from a published population meta-analysis of ultrasonographic corneal pachymetry <sup>21</sup>. For every 50&micro;m difference in CCT from the reference value of 545 &micro;m, the linear correction scale added or substracted 2.5 mmHg. A correction factor (CF) of 2.5 mmHg was used of each eye&nbsp; <sup>20</sup>. The recorded IOP measurement was made according to the following linear formula:&nbsp; Adjusted IOP (aIOP) = measured IOP - (CCT-454)/50 &times; 2.5 mmHg <sup>20</sup>.</p>\r\n				<p>\r\n					The recorded IOP measurements were then adjusted for CCT using 545&micro;m as the reference value (from a review of 80 studies<sup>20</sup>using ultrasound pachymetry, the mean &plusmn; SD CCT was 544 &plusmn; 34 &micro;m)<sup>21</sup>. The IOP results were corrected using a linear algorithm which is non-validated at the time of study. The linear correction scale added or substracted 2.5 mmHg for every 50&micro;m difference in CCT from the reference value of 545 &micro;m. A correction factor (CF) of 2.5 mmHg was used<sup>21</sup>. Adjustment of IOP was made according to the following linear formula:&nbsp; Adjusted IOP (aIOP) = measured IOP - (CCT-454)/50 &times; 2.5 mmHg <sup>21</sup>.</p>\r\n				<p>\r\n					In order to minimise error, measurements were performed by the same researcher and the average of three intraocular pressure were taken using the same equipment or machine and the average of two out of three highly correlating central corneal thickness readings with the same machine. All measurements were taken during the daytime only to reduce the effect of diurnal variation.</p>\r\n				<p>\r\n					Statistical analyses in this study were performed with the Statistical Package for Social Sciences (SPSS) version 19. Paired t-test was used to compare the IOP, CCT and body weight measurement before and after HD. Pearsons correlation test was used to investigate possible correlations between changes in IOP, CCT, and total body volume loss after HD. Possible correlations between IOP and CCT, CCT and changes in body weight, and IOP and changes in body weight were also investigated by Pearson correlation test. A level of p &lt; 0.05 was considered statistically significant.</p>\r\n				<p>\r\n					<strong>Results</strong></p>\r\n				<p>\r\n					A total of 50 eyes from 50 patients were recruited. Only one eye was used for analysis. The right eye was selected unless it did not meet the inclusion criteria or the CCT reading was not reliable, then the fellow eye was used. &nbsp;In total, 45 (90%) right eyes were used. The majority of participants were in-patients. All the patients were clinically stable and free of acute illness. Table 1[<strong>Table 1]</strong> records the demographical data of the subjects. Females outnumbered males and there were more diabetics (64%).</p>\r\n				<p>\r\n					Majority of patients had been on HD for less than 11 months [<strong>Table 2]</strong>. During HD, none of the patients had symptoms or signs of acute raised IOP, such as blurring of vision, eye pain or headache.</p>\r\n				<p>\r\n					The mean IOP before and after HD were 14.76 &plusmn;3.35mmHg and 15.08 &plusmn;3.36mmHg respectively. The differences in the IOP before and after HD were not statistically significant, (paired t test, p = 0.798<strong>) [Table 3]. </strong>The mean CCT decreased significantly after HD <strong>[Table 3].</strong> The mean decrease in CCT was 4.5 &plusmn; 10.6 &micro;m. Although the corneal thickness decreased after HD in 64% of eyes, it was equal to or more than the pre-HD thickness in 36% of eyes. There was a significant decrease in body weight from before haemodialysis (mean 63.0 &plusmn; 15.5 kg) to after haemodialysis (mean 61.3 &plusmn; 15.0 kg) <strong>[Table 3].</strong></p>\r\n				<p>\r\n					There was no significant correlation between the change in CCT and that of change in IOP as well as aIOP (r = -0.051, p = 0.724; r = 0.237, p = 0.098) before and after HD. <strong>[Figure 1]</strong></p>\r\n				<p>\r\n					Corneal thickness decreased significantly after HD.&nbsp; When the changes in CCT measurement were plotted against the changes in total body volume after HD, no significant correlation was found between the two parameters (r = - 0.015, p = 0.920) <strong>[Figure 2]</strong>.</p>\r\n				<p>\r\n					No correlation was found between the changes in IOP with changes in total body volume loss after HD (r = 0.191, p = 0.185) <strong>[Figure 3]</strong>. Similarly, there was also no correlation between the changes in aIOP with changes in total body volume after HD (r = - 0.188, p = 0.190).&nbsp; <strong>[Figure 4]</strong>.</p>\r\n				<p>\r\n					&nbsp;</p>\r\n				<p>\r\n					<strong>Discussion</strong></p>\r\n				<p>\r\n					According to the 17<sup>th</sup> report of the Malaysian Dialysis registry 2009<sup>1</sup>, the prevalence of dialysis is slightly higher in males with the percentage of 55% so the majority of female respondents is unusual for our general population. The majority of patients involved in this study were of Malay and Chinese parentage which reflected the ethnic distribution of the local district population. The mean age of the study group was 54.3 &plusmn; 11.5 years, consistent with the data from the Malaysian registry report, which found more than half of the patients were at least 55 years old<sup>1</sup>. Diabetic nephropathy was reported to be the commonest cause of primary renal disease in Malaysia followed by idiopathic (30%), hypertension (7%) and glomerulonephritis (4%)<sup>1</sup>.</p>\r\n				<p>\r\n					&nbsp;</p>\r\n				<p>\r\n					Sitprija et al<sup>7</sup> who were the first to study the relationship between plasma osmolarity and IOP during HD, reported that a rapid decrease in plasma osmolarity between body compartments markedly increased the IOP. This mechanism is analogous to cerebral oedema which occurs in the disequilibrium syndrome. The mechanism proposed was that the movement of water from the plasma into the tissue, namely the ciliary bodies, leads to an increased aqueous production and raised IOP <sup>7</sup> . This fact was supported by Burn et al<sup>16</sup> and Tawara et al<sup>11</sup> who also included the influence of aqueous outflow facility in this theory. They found that the rise in IOP is small provided that the eye-outflow system is normal. However, in patients with eye-outflow obstruction due to anterior synechiae or narrow angle between the iris and the cornea, IOP may rise sharply during HD and lead to acute glaucoma. This was not seen in any of our patients.</p>\r\n				<p>\r\n					Our study demonstrated that there was no statistically significant change in IOP before and after HD. The mean IOP increased from 14.76 &plusmn; 3.35 mmHg pre-dialysis, to 15.08 &plusmn; 3.66 mmHg post-dialysis. A number of other reports have documented a similar observation <sup>13, 15 </sup>. On the other hand, our findings contradicted those of Dinc et a.l<sup>22</sup>, who reported a significant decrease in IOP after HD, with a mean reduction of 1.3 &plusmn; 2.4 mmHg. This difference may be explained by the fact that changes in body fluid osmolarity are not the only factor responsible for IOP rise during HD. Nevertheless, it is safe to conclude that any specific observed trend of IOP change with HD cannot be generalized, due to the dynamic nature of IOP and multitude of physiological factors affecting it.</p>\r\n				<p>\r\n					Another theory proposed by Tokuyama et al.<sup>23</sup>suggests that the change in IOP during HD was correlated with the change in plasma colloid osmotic pressure and the change in body weight. They suggested that plasma colloid osmotic pressure is instrumental in the hydrodynamic changes that occur during HD.</p>\r\n				<p>\r\n					In our study, the highest rise of IOP after HD was 7.0 mmHg and the highest decrease in IOP was 6 mmHg. (Figure 4) The wide discrepancy of IOP changes in our study clearly explained that the underlying mechanisms are varied, and other factors might also play a role, even though it was not studied in our patients. We believe that eyes with obstructed aqueous outflow pathways could have been included in our study population, which can affect the outcome of this particular study.</p>\r\n				<p>\r\n					There were a few differences in the methodology between our study and that of the other studies.&nbsp; In our study, patients with systemic illnesses such as hypertension and diabetes mellitus were included, whereas Pelit et al.<sup>24</sup>excluded these patients. The reason for this was a Humphrey visual field test was included in their study, which required full attention from the patients. Their attention span can be affected by the multiple drugs used for the systemic illnesses. However, we feel that this effect was minimal given that the patients were all able to comply with the investigations. Patients with any retinopathy including diabetic and hypertensive retinopathy were also excluded in Dinc et al.&rsquo;s study<sup>22</sup>.</p>\r\n				<p>\r\n					There is sparse Malaysian data on ocular changes following HD.&nbsp; To date, there is only one published study conducted by Samsudin et al.<sup>1</sup><sup>2</sup>, which reports the IOP changes following HD namelyIOP decreased significantly in eyes with occludable angle, but changes were not significant in the non-occludable angle group.</p>\r\n				<p>\r\n					With respect to corneal changes, Costagliola et al.<sup>10</sup> did not find any differences in pachymetric measurements in chronic renal failure (CRF) patients 1 hour before and after HD. Our study also investigated the acute effect of HD on corneal thickness and hypothetised that there would be a decreased corneal thickness after HD paralleling decrease in total body volume loss. We demonstrated a significant decrease in CCT in&nbsp; study eyes after HD with a mean of 4.5&plusmn; 10.6 &micro;m similar to Dinc&rsquo;s study<sup>22</sup>&nbsp; indicating that large volume changes caused by HD indeed results in corneal deturgescence . Unfortunately, the inter-test variability data of our CCT measuring device was not available,and it is possible that the change observed may not be significant. However, while Dinc et al found a positive correlation between changes in total body fluid and CCT, our study could not demonstrate the correlation as such. This could be due to the fact that other factors might also play a role in the change of the corneal thickness following HD. Furthermore, in our study, 18 eyes (36%) showed increase in CCT after HD. We speculate that, this increase might be the result of permanent endothelial damage caused by uraemic toxins which could not be reversed by HD. The effect of uraemic toxins in causing endothelial damage, leading to polymegathism and pleomorphism in patients with CRF has been reported in previous study<sup>2</sup></p>\r\n				<p>\r\n					One patient in our study had pre-HD IOP of 24 mmHg which decreased to 18 mmHg after HD. The CCT before and after HD for this particular patient was 641.67 &micro;m and 625.3 &micro;m respectively. It clearly showed the relation between CCT and IOP. IOP may be overestimated in thick corneas and underestimated in thin corneas when using Goldmann tonometry. In our study, a correction factor for aplanation tonometry readings of&nbsp; 2.5 mmHg for every 50 &micro;m difference from CCT from the reference value of 545 &micro;m were used giving&nbsp; aIOP value of each patient.<sup>21</sup>The disadvantage of a linear correction factor includes disregarding the effects of other factors including corneal hysteresis. We found a significant decrease in CCT in study eyes after HD. As a consequence of this consideration, we investigate the correlation between the changes in CCT and IOP after HD. Nevertheless, we could not demonstrate a significant correlation between the change in CCT and that of change in IOP as well as aIOP<strong>.</strong></p>\r\n				<p>\r\n					In our study, mean body weight was found to be 63.0 &plusmn; 15.5 kg before haemodialysis, which decreased to 61.3 &plusmn; 15.0 kg after haemodialysis. The decline in body weight was statistically significant, with a mean change of 1.64 kg &plusmn; 1.23 (p = &lt;0.001) as expected. Animal studies showed that a rise in IOP occurred in association with increase in body fluid content, increase in pCO<sub>2</sub>, and a drop in plasma osmolality but was not related to pH<sup>25</sup>.&nbsp; In agreement with a previous study<sup>22</sup>, our data showed that the rise in IOP was not correlated with an increase in total body volume loss (as indicated by weight changes). In fact, many patients showing a rise in IOP had an associated decrease in body fluid content.&nbsp;</p>\r\n				<p>\r\n					There were several limitations in our study. One is the small numbers of each ethnic group. It is possible there is a difference in aqueous outflow between groups of different ethnicity. For instance, there is a higher incidence of angle closure in those of Chinese ethnic group. Another study weakness is the absence of formal testing of refractive errors. Longer axial lengths are associated with a thinner CCT. Hence this study looked at the changes in CCT.</p>\r\n				<p>\r\n					Another limitation of this study was the CCT measurement was taken by the same observer, giving rise to a potential intraobserver bias. Futhermore, the inter-test variability data of our CCT measuring device was not available, therefore the accuracy of the central corneal thickness measurements cannot be determined.</p>\r\n				<p>\r\n					Our study also included diabetic patients who are known to be at risk of developing glaucoma. The status of diabetic retinopathy was not documented in this study. Patients who have proliferative diabetic retinopathy with secondary rubeotic glaucoma could have been recruited, which can affect the outcome. Another limitation is the study did not take into account the plasma osmolarity change or duration of HD. Gonioscopy was also not performed.</p>\r\n				<p>\r\n					In conclusion, our study provides baseline values of IOP and CCT changes in a population of Malaysian patients undergoing HD. Based on this data, patients undergoing HD do not appear to be susceptible to severe fluctuations of IOP. Clinical application of the study includes assessment of patients with glaucoma where CCT is one of the main parameters in estimating IOP.</p>\r\n				<p>\r\n					<strong>References</strong></p>\r\n				<p>\r\n					1.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Lim Y, Lim T, Lee D. 17th Report of the Malaysian Dialysis and Transplant registry. National Renal Registry,Malaysian Society of Nephrology:Kuala Lumpur,2009.</p>\r\n				<p>\r\n					2.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Diaz-Couchoud P, Bordos F, Garcia J, et al. Corneal disease in patients with chronic renal insufficiency undergoing haemodialysis. Cornea. 2001;20:695-702.</p>\r\n				<p>\r\n					3.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Tay E, Andreou P, Graham E. Anterior Iscahemic Optic Neuropathy (AION) Associated with Post Dialysis Hypotension. Neuro-Ophthalmology. 2009;33:168-73.</p>\r\n				<p>\r\n					4.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Romero C, Rai M, Lowder C, et al. Endogenous endophthalmitis:case report and brief review. Am Fam Physician. 1999(60):510-4.</p>\r\n				<p>\r\n					5.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Choong Y, Menage M. Symptomatic acute raised IOP following hemodialysis in a patient with end stage renal failure. Br J Ophthalmol. 1998;82(1342-50).</p>\r\n				<p>\r\n					6.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Masuda H, Shibuya Y, Ohira A. Markedly increased unilateral intraocular pressure during hemodialysis in a patient with ipsilateral exfoliative glaucoma. Am J Ophthalmol. 2000;129(534-6).</p>\r\n				<p>\r\n					7.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Sitprija V, Holmes J, Ellis P. Intraocular pressure change during artificial kidney therapy. Arch Ophthalmol. 1964;72:626-31.</p>\r\n				<p>\r\n					8.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Austin N, Klein M, Mishell J, et al. Intraocular pressure during high-flux haemodialysis. Ren Fail. 1990;12:109-12.</p>\r\n				<p>\r\n					9.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Fischur M, Fleischhauer J, Keusch G, et al. Rise in intraocular pressure during haemodialysis in a patient with reduced outflow facility. Br J Ophthalmol. 2007;91:1091-3.</p>\r\n				<p>\r\n					10.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Costagliola C, Mastropasqua L. The influence of hemodialysis on intraocular pressure: III. Aqueous humour dynamics and tissue hydration. Ann Ophthalmol. 1991;23:31-4.</p>\r\n				<p>\r\n					11.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Tawara A, Kobata H, Fujisawa K, et al. Mechanism of intraocular pressure elevation during hemodialysis. Curr Eye Res. 1998;17:339&ndash;47.</p>\r\n				<p>\r\n					12.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Samsudin A, Mimiwati Z, Soong T, et al. Effect of haemodialysis on intraocular pressure. Eye. 2009:1-4.</p>\r\n				<p>\r\n					13.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Tokuyama T, Ikeda T, Ishikawa H, et al. Marked decrease in intraocular pressure in a neovascular glaucoma patient during hemodialysis. Jpn J Ophthalmol. 1997;41:101&ndash;3.</p>\r\n				<p>\r\n					14.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Doshiro A, Ban Y, Kobayashi L, et al. Intraocular pressure change during haemodialysis. Am J Ophthalmol. 2006;142:337-9.</p>\r\n				<p>\r\n					15.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Ramsell J, Ellis P, Paterson C. Intraocular pressure changes during hemodialysis. Am J Ophthalmol. 1971;72:926-30.</p>\r\n				<p>\r\n					16.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Burn R. Intraocular pressure during haemodialysis. Br J Ophthalmol. 1973;57:511-3.</p>\r\n				<p>\r\n					17.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Gafter U, Pinkas M, Hirsch J. Intraocular pressure in uremic patients on chronic hemodialysis. Nephron. 1985;40:74-5.</p>\r\n				<p>\r\n					18.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Leiba H, Oliver M, Shimshoni M, et al. Intraocular pressure fluctuations during regular hemodialysis and ultrafiltration. Acta Ophthalmol. 1990;68:320-2.</p>\r\n				<p>\r\n					19.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Levy J, Tovbin D, Lifshitz T, et al. Intraocular pressure during haemodialysis: a review. Eye. 2005;19(1249-1256).</p>\r\n				<p>\r\n					20.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Shih C, Graff Zivin J, Trokel S. Clinical significance of central corneal thickness in the management of glaucoma. Arch Ophthalmol. 2004;122:1270-5.</p>\r\n				<p>\r\n					21.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Doughty M, Zaman M. Human corneal thickness and its impact on intraocular pressure measures: a review and meta-analysis approach. Survey Of Ophthalmology. 2000;44(5):367-408.</p>\r\n				<p>\r\n					&nbsp;22.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Dinc U, Ozdek S, Aktas Z, et al. Changes in intraocular pressure, and corneal and retinal nerve fiber layer thickness during hemodialysis. Int Ophthalmol. 2010;30:337-40.</p>\r\n				<p>\r\n					23.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Tokuyama T, Ikeda T, Sato K. Effect of plasma colloid osmotic pressure onintraocular pressure during haemodialysis. Br J Ophthalmol. 1998;82:751-3.</p>\r\n				<p>\r\n					24.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Pelit A, Zumrutdal A, Akova Y. The effect of hemodilysis on visual field test in patients with chroic renal failure. Current Eye Research. 2003;26(5):303-6.</p>\r\n				<p>\r\n					25.&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Sitprija V, Holmes J, Ellis P. Changes in intraocular pressure during hemodialysis. Ivestigative Ophthalmology. 1964:273-84.</p>\r\n				<p>\r\n					&nbsp;</p>\r\n				<p>\r\n					&nbsp;</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n', 1),
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(980, 975, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey is the natural sweet substance produced by honey bees from the nectar of blossomed flowers which is called blossom (floral) or nectar honey and can either be unifloral (nectar from same flower) or mulifloral (nectar of various types of flowers). It can also be found from the secretions of living parts of plants or excretions of plant sucking insects on the living parts of plants which is called honeydew (non floral) honey. The collected nectar and honeydew by the honey bees are transformed and combined with specific substances from the bees, and stored in the honey comb to ripen and mature (Codex Alimentarius Commission 2001).</div>\r\n<div>\r\n	Honey production and its use have a long and varied history. It has been traditionally used as food and medicine products since the earliest times by primeval humans as well as the Ancient peoples of Egypt, China, India, Greece and Rome. The first written reference about honey was on a clay tablet of the Sumerians in the Euphrates valley dating back to 2100-2000 BC, which mentioned the use of honey as a drug and an ointment (Crane 1975).&nbsp;</div>\r\n<div>\r\n	In the first compendium of ancient Chinese Medicine Shennong, the year 200 AD, it was mentioned in a written form for the first time that there were many prescriptions and medical indications which contain honey (Bogdanov 2010a). Likewise, in ancient India, Ayurvedic medicine (a Hindu system of traditional medicine native to India and a form of alternative medicine) uses honey for many purposes.&nbsp;</div>\r\n<div>\r\n	According to the bible, King Solomon has said: &ldquo;Eat honey my son, because it is good&rdquo; (Old Testament, proverb 24:13). In Islam, there is an entire surah in the Al-Qur&rsquo;an called Al-Nahl (the bee) that narrates the goodness of honey: &ldquo;And your Lord inspired to the bee, take for yourself among the mountains, houses, and among the trees and [in] that which they construct. Then eat from all the fruits and follow the ways of your Lord laid down [for you].There emerges from their bellies a drink, varying in colors, in which there is healing for people. Indeed in that is a sign for a people who give thought&rdquo; (Al-Qur&rsquo;an, Al-Nahl 16:68-69) and also Allah says that &ldquo;In paradise will be rivers of pure honey&rdquo; (Al-Qur&rsquo;an, Muhammad 47:15). Prophet Muhammad (peace be upon him) strongly recommended honey for the healing purposes and commented on its good values 1400 years ago (Sahih Bukhari).&nbsp;</div>\r\n<div>\r\n	In the past decade, the benefits of honey become more focused on its medicinal properties such as antibacterial, antioxidant, anti-inflammatory and anticancer properties (Al-Mamary et al. 2002, Ghashm et al. 2010, Mohd Nasir et al. 2010, Wen et al. 2012). An alternative medicine branch called &ldquo;Apitherapy&rdquo; has developed offering treatments based on honey and other bee products against many diseases (Bogdanov 2009a).&nbsp;</div>\r\n<div>\r\n	In this review, we highlighted the components present in honey, its therapeutic properties beneficial to human health as well as its nutritional value based on scientific evidences.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MODE OF PROCESSING</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey can be categorized regarding the mode of processing given below (Joshi 2008):</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Squeezed honey: a traditional method of honey extraction, which involves squeezing the honey combs.</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Drained honey: honey is obtained by draining decapped and broodless combs.</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Extracted honey: honey is obtained by centrifuging decapped honey combs which is mainly produced by beekeepers who manage bees in moveable comb hives.</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Pasteurized honey: honey that has been heated in a pasteurization process (71.7 &deg;C or higher). Pasteurization reduces the moisture level, destroys yeast cells and liquefies any microcrystals in the honey. However, excessive heat exposure also results in product deterioration, as it increases the level of hydroxymethylfurfural (HMF) and reduces enzyme activity (e.g. diastase). The heat also affects appearance (darkens the natural honey color), taste and fragrance (Manley 1985).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CONSISTENCY AND APPEARANCE</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey can be classified also according to its consistency and appearance (Joshi 2008):</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Liquid honey: the honey is thinner or thicker in consistency and also free of any visible crystals.</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Crystallized honey: honey is completely granular or solidified; it is also called &ldquo;granulated honey&rdquo;.</div>\r\n<div>\r\n	&bull;<span class="Apple-tab-span" style="white-space:pre"> </span>Partially crystallized honey: a mixture of liquid and crystallized honey.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	COLOR OF HONEY</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Color of honey can be categorized into seven categories with Pfund color scale of 0 to more than 114 mm, as shown in Table 1 (The National Honey Board 2010). The color of honey partly reflects the content of pigments with antioxidant properties &nbsp;such ascarotenoids and flavonoids (Khalil et al. 2011). In addition, color intensity of honey may be considered as a good indication of its antioxidant capacity (Pilijac-Zegarac et al. 2009).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Table 1:<span class="Apple-tab-span" style="white-space:pre"> </span>The color classification in honey.</div>\r\n<div>\r\n	Color<span class="Apple-tab-span" style="white-space:pre"> </span>Pfund scale (mm)<span class="Apple-tab-span" style="white-space:pre"> </span>Optical Density</div>\r\n<div>\r\n	Water White<span class="Apple-tab-span" style="white-space:pre"> </span>&lt; 8<span class="Apple-tab-span" style="white-space:pre"> </span>0.0945</div>\r\n<div>\r\n	Extra White<span class="Apple-tab-span" style="white-space:pre"> </span>09 - 17<span class="Apple-tab-span" style="white-space:pre"> </span>0.189</div>\r\n<div>\r\n	White<span class="Apple-tab-span" style="white-space:pre"> </span>18 - 34<span class="Apple-tab-span" style="white-space:pre"> </span>0.378</div>\r\n<div>\r\n	Extra Light Amber<span class="Apple-tab-span" style="white-space:pre"> </span>35 - 50<span class="Apple-tab-span" style="white-space:pre"> </span>0.595</div>\r\n<div>\r\n	Light Amber<span class="Apple-tab-span" style="white-space:pre"> </span>51 - 85<span class="Apple-tab-span" style="white-space:pre"> </span>1.389</div>\r\n<div>\r\n	Amber<span class="Apple-tab-span" style="white-space:pre"> </span>86 - 114<span class="Apple-tab-span" style="white-space:pre"> </span>3.008</div>\r\n<div>\r\n	Dark Amber<span class="Apple-tab-span" style="white-space:pre"> </span>&gt; 114<span class="Apple-tab-span" style="white-space:pre"> </span>-----</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	COMPOSITION OF HONEY</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey is a supersaturated solution of sugars, mainly composed of fructose and glucose, containing also other important compounds such as minerals, proteins, enzymes, free amino acids, vitamins and many phenolic compounds (Alvarez-Suarez et al. 2010) as well as some organic acids and volatile substances (Perez et al. 2002, Mato et al. 2003).&nbsp;</div>\r\n<div>\r\n	Composition of honey is summerised in Table 2 (The National Honey Board 2010). The composition of honey is rather variable and primarily depends on the botanical origin and the floral source (Persano-Oddo &amp; Piro 2004). However, certain external factors also play a role such as seasonal, environmental, processing, handling, storage, and climatic conditions (Lachman et al. 2010).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Table 2:<span class="Apple-tab-span" style="white-space:pre"> </span>The composition commonly found in honey.</div>\r\n<div>\r\n	Composition<span class="Apple-tab-span" style="white-space:pre"> </span>Range</div>\r\n<div>\r\n	Fructose/Glucose ratio<span class="Apple-tab-span" style="white-space:pre"> </span>0.76 - 1.86</div>\r\n<div>\r\n	Fructose, %<span class="Apple-tab-span" style="white-space:pre"> </span>30.91 - 44.26</div>\r\n<div>\r\n	Glucose, %<span class="Apple-tab-span" style="white-space:pre"> </span>22.89 - 40.75</div>\r\n<div>\r\n	Mineral, %<span class="Apple-tab-span" style="white-space:pre"> </span>0.020 - 1.028</div>\r\n<div>\r\n	Moisture, %<span class="Apple-tab-span" style="white-space:pre"> </span>13.4 - 22.9</div>\r\n<div>\r\n	Reducing sugar, %<span class="Apple-tab-span" style="white-space:pre"> </span>61.39 - 83.72</div>\r\n<div>\r\n	Sucrose, %<span class="Apple-tab-span" style="white-space:pre"> </span>0.25 - 7.57</div>\r\n<div>\r\n	pH<span class="Apple-tab-span" style="white-space:pre"> </span>3.42 - 6.10</div>\r\n<div>\r\n	Total acidity, meq/kg<span class="Apple-tab-span" style="white-space:pre"> </span>8.68 - 59.49</div>\r\n<div>\r\n	Protein, mg/100g<span class="Apple-tab-span" style="white-space:pre"> </span>57.7 - 567</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CARBOHYDRATES &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey is mainly made up of carbohydrates (82.3%) which is a highly complex mixture of sugars. The major sugars in honey are monosaccharides consisting of fructose (38%) and glucose (31%) (Lawal et al. 2009, Alvarez-Suarez et al. 2010) which are responsible for most of the physical and nutritional charactristics of honey (Sato &amp; Miyata 2000). Some of the disaccharides have also been identified in honey such as sucrose, which is composed of fructose and glucose linked together, maltose, kojibiose, turanose, isomaltose, and maltulose, which comprise less than 8% of the composition of honey. Honey also contains trisaccharides such as melezitose and raffinose (Cotte et al. 2004, The National Honey Board 2010).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MOISTURE</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The moisture in honey or water content is the most important component and related to honey quality especially concerning the risk of spoilage due to fermentation and granulation or crystallization during storage (Finola et al. 2007). In general, moisture content in ripe honey is less than 20% while under very humid or tropical conditions the moisture can be more than 20% (Bogdanov 2010b) but only honeys with less than 18% moisture content can be stored with little or no risk of fermentation.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	PROTEINS, ENZYMES AND AMINO ACIDS &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey naturally contains small amount of enzymes that are introduced into honey by the bees during various phases of the honey manufacturing process. The three main honey enzymes are amylase, which decomposed starch or glycogen into smaller sugar units dextrin and maltose, invertase (saccharase, &alpha;-glucosidase) which converts sucrose to fructose and glucose as well as glucose oxidase which produce hydrogen peroxide and gluconic acid from glucose in the presence of water (Bansal et al. 2005, Bogdanov et al. 2008). However, other enzymes are generally present in lesser amounts such as catalase and acid phosphatase (Weston 2000, The National Honey Board 2010).</div>\r\n<div>\r\n	The amino acids identified in honey from different botanical and geographical origin are: glutamic acid, aspartic acid, glutamine, histidine, glycine, threonine, &beta;-alanine, arginine, &alpha;-alanine, &gamma;-aminobutyric acid, proline, tyrosine, valine, methionine, cysteine, isoleucine, leucine, tryptophan, phenylalanine, ornithine and lysine (Iglesias et al. 2004, Perez et al. 2007). &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	VITAMINS AND MINERALS</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey contains varying amounts of vitamins, minerals and trace elements. The vitamins content in honey is low. However, vitamins such as phyllochinon (K), thiamin (B1), riboflavin (B2), niacin (B5) and pyridoxine (B6) as well as vitamins A, E and C have been reported to be present in honey (Al-Waili 2003, Bogdanov et al. 2008). The minerals content of honey depends mainly on the botanical and geographical origin of honey (Bogdanov et al. 2007), however; light color blossom honeys have a lower mineral content than dark color honeys (Bogdanov 2009b).&nbsp;</div>\r\n<div>\r\n	Minerals commonly found in honey are potassium, aluminium, cadmium, barium, nickel, chromium, cobalt, antimony, calcium, iron, zinc, mangnesium, copper, sodium, manganese and selenium (Stocker et al. 2005, Bogdanov 2009b). &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	VOLATILE SUBSTANCES&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The main volatile compounds in honey have their origins in different chemical families such as alcohols, ketones, aldehydes, acids, esters and terpenes (Zhou et al. 2002) and these aroma compounds are present in honey at very low concentrations (Cuevas-Glory et al. 2007). The volatile substances mainly derived from the nectar sources give distinctive flavours and aroma to honey.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	POLYPHENOLIC COMPOUNDS&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Polyphenolics are another important group of compounds regarding the appearance and the functional properties of honey. Polyphenols in honey contribute significantly to the antioxidant capacity of honey and may also be used as biomarkers for the origin of honey (Tomas-Barberan et al. 2001). The polyphenols present in honeys are flavonoids such as hesperetin, kaempferol, quercetin and chrysin, and phenolic acids: p-coumaric, abscisic, ellagic, gallic and ferulic acids (Yaoa et al. 2005, Kenjeric et al. 2007).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	HYDROXYMETHYLFURFURAL (HMF)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Hydroxymethylfurfural (HMF) or 5-hydroxymethyl-2-furaldehyde, is a heterocyclic aldehyde (Figure 1) (Bogdanov 2009b) which forms as a result of hexoses degradation in an acidic environment (Semkiw al. 2010). The presence of simple sugars such as glucose and fructose with many acids in honey is a favorable condition for the production of this substance (Khalil et al. 2010).&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Figure 1:<span class="Apple-tab-span" style="white-space:pre"> </span>Chemical structure of Hydroxymethylfurfural (HMF).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	HMF value is almost absent or very low in fresh honey and its concentration increases as a result of heating processes, long-term storage or stored in non-adequate conditions, use of metallic containers and adulterated with invert syrup. Thus, HMF measurement is used to evaluate the freshness and quality of honey (Zappala et al. 2005, Ajlouni &amp; Sujirapinyokul 2010). The Codex Alimentarius (2001) and International Honey Commission (2002) set the maximum concentration of HMF to 40 mg/kg for honey from non-tropical region and 80 mg/kg for honey from tropical regions. Honey is thus best consumed within six months of purchase to reduce the production of HMF.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NUTRITIONAL BENEFITS OF HONEY</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey is one of the oldest and best-loved sweetening agents for foods from olden days until now (Al-Qassemi &amp; Robinson 2003). It is an important source of carbohydrates and the only widely available sweetener which provides strength and energy to our body (Benefits of Honey 2012).</div>\r\n<div>\r\n	The carbohydrates in honey are easily digested and quickly transported into the blood and can be utilized for energy requirements by the human body. The glucose in honey is absorbed by the body quickly and gives an immediate energy boost, while the fructose is absorbed more slowly providing sustained energy. It is known that honey has also been found to keep levels of blood sugar fairly constant compared to other types of sugar. Thus, honey is particularly recommended for children and sportsmen because it can help to improve on the efficiency of the energy system of the elderly and invalids (Alvarez-Saurez et al. 2010, Benefits of Honey 2012).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	THERAPEUTIC PROPERTIES OF HONEY</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Gastroenterology&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Infections of the intestinal tract are common throughout the world and affect people of all ages. The use of honey for prevention and treatments of gastrointestinal disorders such as peptic ulcers, gastritis and gastroenteritis has been reported in various publications around the world (Bogdanov et al. 2008). Honey has antimicrobial activity against bacteria that can cause diarrhea especially the ones that are caused by E. coli (Adebolu 2005) and against many enteropathogenic organisms including those of the Salmonella and Shigella species (Molan 2001a).</div>\r\n<div>\r\n	Helicobacter pylori (H. pylori) infection is probably one of the most common bacterial infections worldwide and complications resulting from this infection caused gastritis, gastric and duodenal ulcers (Sherif et al. 2004, Tiwari et al. 2005). Honey derived remedies comprise a potential source of new compounds that may be useful in the management of H. pylori infections (Manyi-Loh et al. 2010). A study carried out by Ndip et al. (2007) on four selected honeys (Mountain, Manuka, Capillano and Eco) was found to exhibit antibacterial activity against H. pylori associated gastrointestinal disease.&nbsp;</div>\r\n<div>\r\n	Studies of the honey action against peptic ulcers experimentally induced in rats by indomethacin (an aspirin-type anti-inflammatory drug) and alcohol have shown that it has a dose-dependent effect protecting the stomach from ulceration being caused by indomethacin and alcohol (Ali &amp; Al-Swayeh 1997, Gharzouli et al. 2002).&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Cardiovascular Health</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Yaghoobi et al. (2008) studied the effect of honey in normal and overweight persons with higher risk for cardiovascular disease. The normal persons received 70 g of sucrose while cardiovascular disease patients received 70 g of honey, for 30 days. The effect of natural honey was investigated on total cholesterol, LDL-C, HDL-C, triacylglyceride, CRP, fasting blood glucose (FBG) and body weight in overweight individuals. The results showed that consumption of natural honey reduces cardiovascular risk factors, by modulating the plasma levels of total cholesterol, low-density lipoprotein cholesterol (LDL-C), high-density lipoprotein cholesterol (HDL-C), triacylglyceride, homocysteine and CRP.&nbsp;</div>\r\n<div>\r\n	It is most likely the antioxidant polyphenols in honey such as quercetin, acacetin, caffeic acid phenethyl ester, kaempferol and galangin have been reported as promising pharmaceutical drugs in the treatment of cardiovascular diseases (Khalil &amp; Sulaiman 2010).&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Wounds and Burns Healings</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The healing properties of honey have been known since time immemorial and have recently gained recognition from the scientific community (Molan 2000). Honey has been used for the treatment of chronic wounds, burns, methicillin-resistant Staphylococcus aureus (MRSA), diabetic foot ulcers and many other ailments (Visavadia et al. 2008, Simon et al. 2009). Honey accelerates wound healing whether applied topically or administered systemically. An infected wound will not heal unless bacteria are eliminated because bacteria stimulate the inflammatory response which can prevent or slow the process of wound healing. Molan (2002) reported that honey has an inhibitory effect on approximately 60 species of bacteria such as aerobes and anaerobes as well as gram-positive and gram-negative bacteria. Honey creates a moist environment without the risk of bacterial growth and with no adverse effects to slow the healing process. Honey has high viscosity which provides a protective barrier thus preventing cross-infection (Molan 2006, Khoo et al. 2010).&nbsp;</div>\r\n<div>\r\n	Therapeutic effects of honey in burn treatment have been reported to cause rapid healing of wounds with less scarring (Yusof et al. 2007). Molan (2001a) found that the application of New Zealand Manuka honey accelerated healing in acute wounds, chronic ulcers, burns and infected wounds. Zohdi et al. (2004) reported that topical application of Malaysian Gelam honey was effective to treat burn wound in rats and honey in form of hydrogel dressings was able to accelerate wound healing due to burn (Zohdi et al. 2012). In another study, Aljadi et al. (2000) found that combined Gelam honey treatment (topical and oral) offers a distinct advantage to wound healing and may be a useful adjuvant in wound management. Furthermore, Malaysian Tualang honey has better results with regards to its control of Pseudomonas aeruginosa and its wound contraction effects on full-thickness burn wound in rats (Khoo et al. 2010). &nbsp;Zaharil et al. (2011) found that Tualang honey impregnated dressings were as effective as silver impregnated hydrofibre dressings in terms of dressing properties, promotion of wound healing and inflammatory reaction for full thickness wounds in rats.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Antimicrobial and Antiviral Activities</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The major contributor to the antimicrobial activity of honey is hydrogen peroxide (Temaru et al. 2007) which is formed from the oxidation of glucose by glucose oxidase to gluconic acid and hydrogen peroxide when honey is diluted (Bang et al. 2003, Iurlina &amp; Fritz 2005). The varying antimicrobial activity in different types of honey is because of the different concentrations of hydrogen peroxide (Molan 1999).&nbsp;</div>\r\n<div>\r\n	The antimicrobial activity of honey is also attributed by some physical factors such as acidity (low pH) and osmolarity (Hamouda &amp; Abouwarda 2011). The high osmolarity of honey is due to the high content of sugar (over 85% of honey) and these sugars have high affinity for water molecules leaving little or no water to support the growth of different microorganisms such as bacteria and yeast. Consequently, the microorganisms become dehydrated and eventually die (Hamouda &amp; Abouwarda 2011). Honey is acidic with a pH range of 3.2 to 4.5 which is low enough to inhibit the presence and growth of many pathogens (Allen et al. 1991).&nbsp;</div>\r\n<div>\r\n	Honey can inhibit the growth of a wide range of bacteria, fungi, protozoa and viruses (Blair &amp; Carter 2005). It has been shown that honey has a broad-spectrum antimicrobial activity on gram-negative and gram-positive bacteria (Agbaje et al. 2006). Several studies have revealed that honey is effective against Methicillin resistant Staphylococcus aureus (MRSA), &beta;-hemolytic Streptococci and Vancomycin-resistant Enterococci (VRE) (Allen et al. 2000, Kingsley 2001). Furthermore, honey has also been shown to have inhibitory effect on the Rubella virus (Zeina et al. 1996) and Leishmania parasite (Zeina et al. 1997) as well as fungi such as Candida spp. and Trichosporon spp. (Koc et al. 2009).</div>\r\n<div>\r\n	Due to its antibacterial activity, honey ingestion inhibits the growth of bacteria causing caries (Steinberg et al. 1996, Molan 2001b) and might induce a carioprotective effect (Sela et al. 1998). It was reported that Manuka honey (a very potent antimicrobial honey) has a positive effect against dental plaque development and gingivitis (English et al. 2004) and thus a useful substitute of refined sugar in the manufacture of candy (Molan 2001b).&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Anti-tumor Activity&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Many studies have shown that high polyphenols in honey has been attributed for its anti-tumor property (Russo et al. 2004, Jaganathan &amp; Mandal 2009a). Jaganathan &amp; Mandal (2009b) illustrated the ability of the honey to induce apoptosis in human colorectal cancer cells attributed by the high phenolics and tryptophan contents. Additionally, Malaysian Tualang honey was found to have significant anticancer activity against human breast cancer and cervical cancer cell lines (Fauzi et al. 2011) as well as osteosarcoma and oral cancer cell lines (Ghashm et al. 2010). Wen et al. (2012) reported that Malaysian Gelam and Nenas honeys are capable of suppressing the growth of HT 29 colon cancer cells by inducing apoptosis and suppressing inflammation.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Anti-inflammatory Activity</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Inflammation is the response of vascularized living tissue to local injury and &nbsp;plays an important role in various diseases such as rheumatoid arthritis, atherosclerosis and asthma, which all show a high prevalence globally (Hung et al. 2009, Mueller et al. 2010).&nbsp;</div>\r\n<div>\r\n	During an inflammatory response, several pro-inflammatory mediators are released including interleukins (IL-1, IL-6, IL-2 and IL-8), tumor necrosis factor (TNF) and interferon (IFN-&gamma;) as well as cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS) which are suppressed by anti-inflammatory cytokines such as IL-4, IL-10, IL-13, IFN-&alpha; and transforming growth factor (TGF-&beta;), which therefore help to balance the inflammatory response (Hanada &amp; Yoshimura 2002, Goldstein et al. 2006, Mueller et al. 2010). Thus, inhibition of the overproduction of inflammatory mediators, especially pro-inflammatory cytokines (IL-1&beta;, IL-6 and TNF-&alpha;), may prevent or suppress a variety of inflammatory diseases (Kim et al. 2003).</div>\r\n<div>\r\n	Honey has been reported to have anti-inflammatory effect in in vitro and in vivo studies. Kassim et al. (2010a) found that Gelam honey extract inhibited the level of nitric oxide (NO) in LPS-stimulated macrophage RAW264.7 cells as well as honey and its extracts have anti-inflammatory effects by reducing the inflammatory mediators NO and PGE2 in rat paw tissues (Kassim et al 2010b). Moreover, honey ingestion decreased inflammation in an experimental model of inflammatory bowel disease in rats (Prakash et al. 2008). Owayele et al. (2011) reported that honey can inhibit the NO release in carrageenan-induced inflammation in rats.&nbsp;</div>\r\n<div>\r\n	Our research group has demonstrated that Malaysian Gelam honey has a potent in vivo anti-inflammatory effect in an acute inflammation model which was induced by a subplantar injection of 1% carrageenan into the rat right hind paw as shown in Figure 2. Gelam honey can inhibit the production of pro-inflammatory NO, PGE2, TNF-&alpha; and IL-6 in plasma of inflammation-induced rats (Figure 3) (Hussein et al. 2012).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Figure 2: Representative photographs of rats&rsquo; right hind paw (a) before (b) after 5hrs and (c) after 24hrs, of carrageenan-induced paw edema inflammation. Edema formation was shown by arrow ( &nbsp; &nbsp; &nbsp; ).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Figure 3: The effect of Gelam honey on (a) NO, (b) PGE2, (c) TNF-&alpha; and (d) IL-6 production in plasma of carrageenan-induced paw edema in rats. D.W= Distilled water, Carr= Carrageenan, IND= Indomethacin. Data presented as mean &plusmn; S.E.M (n=6).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Reproductive Health</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	There is a traditional belief that honey may enhance fertility and vitality as well as improve male reproductive health. It had been reported that daily treatment of 5% Palestinian honey for 20 days increased spermatogenesis in adult rats (Abdul-Ghani et al. 2008). Abdelhafiz &amp; Muhamad (2008) observed that diluted Egyptian honey and royal jelly had an enhancing effect on sperm motility, particularly in subnormal samples (in vitro study).&nbsp;</div>\r\n<div>\r\n	Mahaneem et al. (2007, 2011) reported that Tualang honey (1.2 g/kg of body weight) significantly increased the percentages of rats achieving intromission and ejaculation as well as increased mating and fertility indexes of male rats exposed to Cigarette Smoke (CS) (Mohamed et al. 2012). Their result suggests that honey has a protective effect against CS-induced impaired sexual behavior and fertility in male rats. Likewise, Asiyah et al. (2011) reported that Malaysian Gelam honey (1.0 ml/100g of body weight) was potentially useful in increasing the fertility of juvenile male rats by increasing sperm motility and number of morphologically normal sperm compared with male rats injected with nicotine.</div>\r\n<div>\r\n	A study by Zaid et al. (2010) on the effects of Tualang honey on female reproductive organs (uterus and vagina) and tibia bone in menopause rats (ovariectomised) showed that Tualang honey has a beneficial effect on menopausal rats by preventing uterine atrophy, increased bone density and suppression of body weight increase. Thus, these findings may suggest honey could be an alternative to hormone replacement therapy (HRT).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CONCLUSION&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Honey is one of the oldest and best sweetening agents for foods over the centuries. It is made up of a vast amount of different compounds that can be of nutritional and health benefits. The main nutrition and health-relevant components are the carbohydrates, especially fructose and glucose, which make it an excellent energy source for human. Besides its main components, honey also contains a great number of other constituents in small and trace amounts including proteins, enzymes, vitamins, minerals and phenolic compounds, producing numerous nutritional and biological effects such as: wound healing, antimicrobial, antiviral, antioxidant, anti-inflammatory and antitumor activities. Thus, the use of honey either as a food or an alternative medicine in the treatment of different disorders is safe, beneficial and free from side effects.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	REFERENCES</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Abdelhafiz, A.T. &amp; Muhamad, J.A. 2008. Midcycle pericoital intravaginal bee honey and royal jelly for male factor infertility. International Journal of Gynecology and Obstetrics 101: 146-149.</div>\r\n<div>\r\n	Abdul-Ghani, A.S., Dabdoub, N., Muhammad, R., Abdul-Ghani, R. &amp; Qazzaz, M. 2008. Effect of Palestinian honey on spermatogenesis in rats. 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Journal of Alternative and Complementary Medicine 2(3): 345-348.</div>\r\n<div>\r\n	Zeina, B., Zohra, B.I. &amp; Al-Assad, S. 1997. The effects of honey on Leishmania parasites: an in vitro study. Tropical Doctor 27(Supp.1): 36-38.</div>\r\n<div>\r\n	Zhou, Q., Wintersteen, C.L. &amp; Cadwallader, R.W. 2002. Identification and quantification of aroma-active compounds that contribute to the distinct malty flavor of buckwheat honey. Journal of Agricultural and Food Chemistry 50: 2016-2021.</div>\r\n<div>\r\n	Zohdi, R.M., Zakaria, Z.A.B., Mustapha, N.M., Yusof, N., &amp; Abdullah, M.N.H. 2004. The effect of topical application of Malaysian honey on burn wound healing. Jurnal Veterinar Malaysia 2004(1-2): 47-50.</div>\r\n<div>\r\n	Zohdi, R.M., Zakaria, Z.A.B., Yusof, N., Mustapha, N.M. &amp; Abdullah, M.N.H. 2012. Gelam (Melaleuca spp.) honey-based hydrogel as burn wound dressing. Evidence-Based Complementary and Alternative Medicine 2012: Article ID 8430</div>\r\n', 1);
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(981, 976, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Tuberculosis is on the rise due to influx of foreign workers into the country. Ocular tuberculosis can either be immunogenic or infective in origin, however, the definitive diagnosis is necessarily challenging. Broadly, the clinical features of ocular tuberculosis give certain clues towards the underlying aetiology, supplemented by different tests. We report an interesting case of seemingly infective ocular tuberculosis in an immunocompetent patient, yet without any signs of inflammation. This case highlights the ocular features, the importance of early management and the difficulties in diagnosis the underlying aetiology of ocular tuberculosis.</div>\r\n<div>\r\n	Case report</div>\r\n<div>\r\n	A 17- year- old male student of Indonesian parentage presented with two weeks history of painless and progressive visual deterioration without photopsia or floaters. He denied any contact with tuberculosis patients or any high- risk behavior. There was no reported back or joint pain, fever or rashes, nor any constitutional symptoms. According to his parents, all immunisation was up to date, including for Bacilli Calmette Gue`rin (BCG).&nbsp;</div>\r\n<div>\r\n	Visual acuity at presentation was 6/36, N48 and 6/60, N- for the right and left eyes respectively. There was no relative afferent pupillary defect, and both the anterior and vitreous chambers were quiet. On funduscopic examination, there was vasculitis involving both arteries and veins with perivascular exudates and signs of branch vein occlusion bilaterally (Figure 1) Blotchy haemorrhages were seen, with cotton wool spots and obvious macular oedema, as evidenced on optical coherence tomography (OCT) (Figure 1).&nbsp;</div>\r\n<div>\r\n	Fluorescein angiography study confirmed large areas of capillary fallout in both eyes and leaking new vessels, consistent with ischaemia secondary to inflammation (Figure 2). Mantoux test was measured to be 15 mm but screening against Human Immunodeficiency Virus (HIV) was negative. Haematological parameters did not show any elevation of the white cell count or its differentials. Erythrocyte sedimentation rate showed increased level of 98 mmol/ hr. otherwise, the test VDRL test was negative and chest X- ray was clear. Patient&rsquo;s family declined to have quantiferron test done.&nbsp;</div>\r\n<div>\r\n	Based on the significant Mantoux test and elevated ESR level, he was diagnosed as ocular TB, although there was no sign of vitritis and anterior chamber inflammation despite being immunocompetent. Full regime of anti- TB therapy was started constituting of rifampin 600 mg,</div>\r\n<div>\r\n	ethambutol 800 mg, isoniazid 300 mg, &nbsp;and pyrazinamide 1000 mg once daily. His macula showed sign of reduction in thickness on follow- up. He also received intensive panretinal laser photocoagulation. Three days following the institution of anti TB, patient was started on 1mg/ kg of oral steroid daily. New vessels showed signs of regression and macula oedema was resolving on both eyes. His visual acuity had improved to 6/ 12 and patient is currently still receiving treatment.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The incident of tuberculosis is on the rise due to influx of foreign workers into the country and also the increasing number of HIV positive patients [Dolin 1994]. Tuberculosis is a bacterial infection which does not only affect the lungs, but may also occur in any other organs, including the eye. Ocular tuberculosis may occur in isolation or as miliary infections from another source [Helm 1993]. The ocular manifestations of TB may either be of immunogenic or infective in origin [Helm 1993], however the definitive diagnosis between these two diagnoses may be challenging. The management of immunogenic ocular TB is different from the infective once, hence the importance of correct diagnosis. Immunogenic TB requires treatment with immunomodulating agents such as steroid, whereas anti- TB medications is necessary for infective cases.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Our patient presented with occlusive retinal vasculitis without other signs of ocular inflammation, despite his immunocompetent status. Generally, immunogenic and infective ocular TB is differentiated by its clinical features [Shree 2006], especially if laboratory results are indefinite. Immunogenic TB tends to present with vascular occlusive features associated with dense vitritis in immunocompetent patient. [Thompson 2005] Infective ocular TB affecting posterior segment usually manifests as multiple choroidal lesions, consistent with the haematologic spread of the bacteria. [Sheu 2001] The Quantiferon-Gold in-tube assay (QFT-IT) is a valuable diagnostic tool to diagnose for infective TB [Rutherford 2010]. However, this may not always be possible for each case due to the finalcial limitation. Furthermore, this particular test does not differentiate between active or latent TB. [Rutherford 2010]&nbsp;</div>\r\n<div>\r\n	Tubeculin- skin (Mantoux) test measures the hypersensitivity reaction to tuberculin.[Surajit 2012] Although the test is neither 100% specific nor 100% sensitive, it is still widely used to screen against TB diseases, especially in high risk populations. Combining both the Tuberculin- skin test and the gold Quantiferon assay may prove to yield better results in diagnosing TB conditions. [Rutherford 2010]</div>\r\n<div>\r\n	The management of ocular TB largely depends on the underlying aetiology. The mainstay of treatment for infective ocular TB (either latent or active) is anti- TB medications, with regular follow- up for monitoring its side effects. Prompt institution of immunosuppressive agent is vision- saving in immune- mediated ocular TB. [Amod 2001] However, this particular case is not a clear cut, hence we opted to err on the safe side and started anti- TB medication, prior to starting the immunosupressive agent. Although patient&rsquo;s condition showed some improvements with the institution of anti- TB, dramatic changes was seen when steroid was started.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Clinical features of ocular TB usually point towards the diagnosis of occlusive retinal vasculitis. However, in some cases the vasculitis and macular conditions cannot be ascertained. Ocular coherence tomography and fluorescein angiogram study provide invaluable additional information. Areas of capillary fallout and leaking new vessels are more apparent on angioram study. Furthermore, macular ischaemia which may occur with or without macular oedema is important as it carries grave visual prognosis.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Ocular tuberculosis may affect individuals despite previous vaccination. The importance of differentiation between immune- mediated and infective TB lies in the management. Fluorescein angiography study and ocular coherence tomography are important diagnostic tools in these cases. High index of suspicion and prompt management is important to save vision, and possibly lives.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	References</div>\r\n<div>\r\n	1.<span class="Apple-tab-span" style="white-space:pre"> </span>PJ Dolin, MC Raviglione, A Kochi . Global tuberculosis incidence and mortality during 1990-2000. Bulletin of the World Health Organization, 1994, 72 (2): 213-220</div>\r\n<div>\r\n	2.<span class="Apple-tab-span" style="white-space:pre"> </span>Helm CJ, Holland GN. Ocular tuberculosis. Surv Ophthalmol. 1993; 38 (3): 229- 256</div>\r\n<div>\r\n	3.<span class="Apple-tab-span" style="white-space:pre"> </span>Shree KK, Chan CC. Mycobacterium-related ocular inflammatory disease: diagnosis and management. Ann Acad Med Singapore 2006; 35(3) :203-9</div>\r\n<div>\r\n	4.<span class="Apple-tab-span" style="white-space:pre"> </span>Thompson MJ, Albert DM. Ocular tuberculosis. Arch Ophthalmol. 2005; 123: 844- 849</div>\r\n<div>\r\n	5.<span class="Apple-tab-span" style="white-space:pre"> </span>Rutherford M, Alisjahbana B, Maharani W, Sampurno H, Ven Crevel R, Hil PC. Sensitivity of the Quantiferon-Gold In-Tube Assay in Sputum Smear Positive TB Cases in Indonesia. PLoS One. 2010; 5(8): e12020.&nbsp;</div>\r\n<div>\r\n	6.<span class="Apple-tab-span" style="white-space:pre"> </span>Sheu SJ, Shyu JS, Chem LM, Chen YY, Chirn SC, Wang JS. Ocular manifestations of tuberculosis. Ophthalmology 2001; 108 (9): 1580- 1585</div>\r\n<div>\r\n	7.<span class="Apple-tab-span" style="white-space:pre"> </span>Surajit N, Acharjya B. Mantoux test and its interpretation. Indian Dermatol Online J. 2012; 3(1): 2&ndash;6</div>\r\n<div>\r\n	8.<span class="Apple-tab-span" style="white-space:pre"> </span>Amod G, Vishali G, Sunil A, Dogra MR, Bambery P. PCR- positive tubercular retinal vasculitis: clinical characteristics and management. Retina 2001; 21(5): 435- 444</div>\r\n', 1),
(982, 977, '<div>\r\n	Case Report</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A 37- year- old policeman, who heavily smoked presented to eye clinic with complaints of sudden and progressive deterioration of the upper half of his right eye visual field. There were no associated floaters or flashes of light, and he denied preceeding ocular trauma.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A month previously, he complained of dry cough with intermittent fever of three weeks duration. X- ray at that time revealed an ill- defined opacity at right midzone with military nodules at both lung fields. (Figure 1) He also admitted to significant weight loss recently. Subsequently, the case was treated as pneumonia and started on oral augmentin 625 mg bidaily for one week. His symptoms did not improve despite completing the antibiotic regime. Respiratory consults advised anti TB therapy on the basis of positive Mantoux test, although Zhiel- Nielson stain and sputum culture were negative. The condition remained the same and he complained of further three kilogram of weight loss within a month, associated with lethargy. Within the same week, the gentleman presented with the ocular symptoms.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	On examination, his visual acuity was counting finger on the affected side, with marked relative afferent pupillary defect. Anterior segment examinations revealed bullous inferior retinal detachment apparent without funduscopy, raising suspicion of masquerade syndrome. (Figure 2) No retinal break was discerned despite careful examination. The contralateral eye was normal.&nbsp;</div>\r\n<div>\r\n	Another repeat X- ray showed new findings of generalized nodules in both lung fields, with right hilar and paratracheal lymphadenopathy. Computed tomography of CT thorax showed a large mass at the right hilum compressing the upper bronchus, and causing secondary collapse of the right upper lobe, wih multiple nodules throughout both lung parenchyma. There were also bilateral hilar and mediastinal lymphadenopathies with extensive metastases to both lungs, adrenal glands, liver, and brain, and right choroid. (Figure 2)</div>\r\n<div>\r\n	Bronchospcopy and biopsy confirmed the diagnosis of poorly differentiated adenocarcinoma of the lung, which was inoperable due to distant metastases. He was planned for combination of radiotherapy and chemotherapy. Unfortunately, patient passed away within two months of his presentation.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Figure 1: &nbsp;Anterior segment photo of dilated right eye shows extensive bullous retinal detachment.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	.`</div>\r\n<div>\r\n	&nbsp; &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Figure 2: CT scans showing multiple nodules throughout lung parenchyma (A), right posterior globe thickening with retinal detachment (B), lesions of brain on MRI (C).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	This case illustrates well the masquerade syndrome, whence an advance stage of lung cancer presented with retinal detachment, signifying distant metastases. In this case, the recognition of symptoms and sign may not be able to save the right eye vision, but it is certainly is life- saving for the patient.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The term &lsquo;masquerade&rsquo; is used to describe uveitides which are not immune mediated in the unusual way, but which have an underlying primary cause.[Zamiri 1997] In this case, metastatic tumour spread from primary small cell lung carcinoma to the right globe caused exudative retinal detachment and minimal vitritis in the eye, mimicking inflammation. Ocular secondaries usually originate from renal and lung carcinoma,[Zamiri 1997] as illustrated in this case. Therefore, our threshold of suspicions for malignancies should be high in atypical ocular presentations of retinal detachment without obvious retinal tear.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The associations between smoking and cancer have always been postulated, yet the exact patho- mechanism is difficult to delineate. P. Yang and colleagues suggest that smokers who develop adenocarcinoma of the lung are as susceptible to the tobacco- containing carcinogens as with the small cell and squamous cell carcinoma.[Young 2002] Furthermore, the same study concluded that the association between adenocarcinoma of the lung and tobacco smoking is stronger than was previously thought.[Young 2002]&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The thickening on the posterior aspect of the right globe was highly likely due to choroidal metastases. Skarin A reported a case of metastatic non- small cell carcinoma that initially presented as choroidal tumour,[Skarin 1999] thus raising a suspicion of primary carcinoma elsewhere. The diagnosis of ocular metastases is based primarily on clinical findings supplemented by imaging studies. A study found that choroids was the sixth most common site for metastatic lung carcinoma, following brain, bone, liver adrenals and lung itself.[Kreusel 2002] Although choroidal metastases is common in advanced lung cancer, [Kreusel 2002] screening for at- risk individuals may have no great benefit due to short survival of affected individuals.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Our patient was given a combination of radiotherapy and chemotherapy, to induce tumour regression. Treated secondary choroidal tumors showed complete regression within 5 months of treatment in 84 % of cases, while serous retinal detachment resolved in 82 % percent of treated metastases within 3.8 months after treatment.[Tsina 2005] In this study, proton beam irradiation led to complications such as keratitis, cataract, radiation papillopathy and radiation maculopathy in 56 % of cases.[Tsina 2005] Although this percentage was quite high, proton beam irradiation allows retention of globe, achieves a high probability of local tumour control and help to avoid pain. [Tsina 2005]</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Reduce visual acuity, although unilateral may have a major impact in one&rsquo;s life, especially in patients who are generally in poor physical condition such as in cancer patients. In a study to look at the safety and efficacy of high dose external beam radiation therapy in patients with choroidal metastases, complete remission of choroidal tumour was achieved in 72 % receiving more than 35.5 Gy and 33 % of patients who received less than that.[Rosette 1998] Visual improvements was also significantly better in the eyes receiving more than 35.5 Gy of radiation dose.[Rosette] Although the dosage was considered high, the improvement in vision following regression of choroidal tumour has a major impact in this group of patients who already have a fatal prognosis.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;Our patient received radiation to the whole brain including the both orbits. Bilateral orbits radiation is recommended to treat subclinical metastases not seen in imaging modalities, and to prevent occurrence of metachoronous metastases.[Rosette 1998]</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Conclusion:</div>\r\n<div>\r\n	Despite the increasing number of pulmonary tuberculosis cases, lung cancer should be foremost in mind, especially in individuals who have history of heavy cigarette smoking. The public should also be made more aware of the devastating complications and carcinogenic effect of smoking. Effects of smoking on the health of the smokers, the family and family economy, and the cost of treatment of various cancers attributed to smoking must be emphasized.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Reference:</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	1.<span class="Apple-tab-span" style="white-space:pre"> </span>Zamiri P, Boyd S, Lightman S. Uveitis in the elderly- is it easy to identify the masquerade? Br J Ophthalmol 1997; 81: 827- 831</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	2.<span class="Apple-tab-span" style="white-space:pre"> </span>P. Yang, Cerhan J R, Vierkant R A, et al. Adenocarcinoma of the lung is strongly associated with cigarette smoking: Further evidence from prospective study of women. Amer J Epidemiology 2002; 156: 1114- 1122</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	3.<span class="Apple-tab-span" style="white-space:pre"> </span>Skarin A. Diagnosis in oncology: choroidal metastases. J Clinical Oncology 1999; 17: 3356- 3359</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	4.<span class="Apple-tab-span" style="white-space:pre"> </span>Kreusel K- M, Wiegel T, Stange M, et al. Choroidal metastases in disseminated lung cancer: frequency and risk factors. Amer J Ophthalmol 2002; 134: 445- 447</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	5.<span class="Apple-tab-span" style="white-space:pre"> </span>Tsina E K, Lane A M, Zacks D N, et al. Treatment of metastaic tumours of the choroid with proton beam irradiation (presented in part at: 35th Panhellinic&nbsp;</div>\r\n<div>\r\n	Ophthalmological Congress, Halkidikim Grecce, June 2002). Ophthalmology 2005; 112: 337- 343</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	6.<span class="Apple-tab-span" style="white-space:pre"> </span>Rosset A, Zografos L, Coucke P, et al. Radiotherapy in choroidal metastases. Radiotherapy and Oncology 1998; 46: 263- 268</div>\r\n', 1),
(984, 979, '<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	<span style="display: none">testing testing<span style="display: none">&nbsp;</span></span></p>\r\n<p>\r\n	<span style="display: none">&nbsp;</span></p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	<span style="display: none">testing testing</span></p>\r\n<p>\r\n	<span style="display: none">&nbsp;</span></p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n', 1),
(992, 968, NULL, NULL),
(993, 987, NULL, NULL),
(997, 991, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Inflammation is part of normal host response to infection and injury. Although inflammation helps clear infection, and along with repair, makes wound healing possible, both inflammation and repair have considerable potential to cause harm. Prolonged presence of inflammatory agents may lead to chronic inflammation.</div>\r\n<div>\r\n	Chronic inflammation can result from various inflammatory agents such as virus, bacteria or antigens that stimulate the activation of inflammatory cytokines (Hussain et al. 2003; Bartsch &amp; Nair 2006). Chronic inflammation processesincrease oxidative and nitrasative stress and lipid peroxidation by inducing oxidant-generating enzymes such as NADPH oxidase, inducible nitric oxide synthase (iNOS) thereby producing excess of diverse reactive oxygen and nitrogen species (ROS and RNS). ROS, RNS and lipid peroxidation products (LPO) are able to cause damage to cell, protein, mRNA, DNA and lipid in both nucleus and mitochondria (Ohshima et al. 2003; Sawa &amp; Ohshima 2006). Lipid peroxides (LPO) such as malondialdehyde (MDA), for example, form adducts with DNA bases. This then leads to alterations in the expressions of signaling molecules (Chung et al. 1996; Bartsch 1999), (West &amp; Marnett 2006), and the functions of enzymes and proteins involved in inflammation such as cyclooxygenase-2 (COX-2) and iNOS (Ohshima 2003).</div>\r\n<div>\r\n	Chronic inflammation is abnormal and the excessive ROS, RNS and LPO produced, facilitate carcinogenesis and other chronic diseases (Hussain et al. 2003; Bartsch &amp; Nair 2005). Several medicinal herbs have been shown to inhibit inflammation as well as antioxidant activity. The antioxidant activity can further protect from oxidative and nitrosative stress damages induced by inflammation process. Compounds from fenugreek spice (Liu et al. 2012), and polyphenols from green tea (Tipoe et al. 2007) have been found to exhibit anti-inflammatory and antioxidant activities. Locally known as pegaga, Centella asiatica is a perennial creeper commonly found in moist areas in tropical and subtropical countries (Brinkhaus et al. 2000). C. asiatica has been used extensively in Ayurvedic and Chinese traditional medicines to treat various ailments such as wounds, ulcer and leprosy (Cheng &amp; Koo 2000; Shetty et al. 2006). The bioactive pentacyclic triterpenoid compounds of C. asiatica, asiaticoside and madecassoside, have been widely studied, and reported to have various biological properties such as wound healing (Shukla et al. 1999), ulcer healing (Guo et al. 2004), anti-apoptosis (Bian et al. 2008), and also immunomodulatory property (Jayathirtha &amp; Mishra 2004).</div>\r\n<div>\r\n	Various methods have been described to prepare C. asiatica extract. However, there is a lack of comparative data on the best method for extracting these bioactive compounds in terms of antioxidant and anti-inflammatory properties. In this study, we compared three different extraction methods for C. asiatica using ethanol, methanol and water and then evaluated the extracts in terms of the phenolic, asiaticosside and madecassoside contents, antioxidant and anti-inflammatory activities.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Plant Material</div>\r\n<div>\r\n	Centella asiatica was bought from a local market in Kuala Lumpur and the sample was deposited at UKM herbarium with voucher number UKMB29984. The whole plant was washed thoroughly with tap water, and dried at 40 0 C until constant weight and coarsely grounded. Pure standards of asiaticoside and madecassoside were obtained from Biopurify (Chengdu, China).</div>\r\n<div>\r\n	Extracts Preparation</div>\r\n<div>\r\n	Sohxlet extraction was used for the preparation of methanol and ethanol extracts. 50g of Centella asiatica coarse plant powder was extracted with 1 litre of either methanol or ethanol for 24 hours and then filtered to collect the extracts. The extracts were then concentrated under vacuum and excess solvent were removed using rotary evaporator.</div>\r\n<div>\r\n	For the preparation of aqueous extract, 50 g of coarse plant powder in 1 litre of distilled water was boiled for 1 hour and the extract filtered. Extract were then freeze-dried.</div>\r\n<div>\r\n	Determination of Asiaticoside and Madecassoside Content</div>\r\n<div>\r\n	The level of asiaticoside and madecassoside in all CA extracts were determined using HPLC. 1mg of ethanol or methanol CA extracts were dissolved in 1ml of methanol. As for the aqueous extract, 1mg of extract was dissolved in ultra pure water. Sample extracts were filtered through 0.2&mu;m filter. A volume of 20&mu;l was injected using an autosampler into the HPLC (Shimadzu, Japan) equipped with a diode array detector. The solvent used for gradient elution were 1% ortophosphoric acid and acetonitrile. The concentration of 1% ortophosphoric acid was set at 5% for the first 5 minutes and then decreased to 80%, 50% and 20% for the next 10, 5 and 7 minutes respectively. The concentration of the 1% ortophosphoric acid was then increased to 50%, 80% and 95% for 5 minutes for each concentration. The analytical column used was ACE C18 with packing material of 5&mu;m particle size at a flowrate of 1.0ml/min at room temperature. Samples were read at 210nm wavelength. Pure standards of asiaticoside and madecassoside concentration range (15&mu;g/ml to 500&mu;g/ml) were used to calibrate standard curve and retention times.</div>\r\n<div>\r\n	Total Phenolic Content Assay</div>\r\n<div>\r\n	Folin-Ciocalteu reagent (diluted 10-fold) was used to determine the total phenolic content of all CA extracts (Velioglu et al. 1998). 100 &mu;l of each sample was mixed with 0.75 ml of Folin-Ciocalteu reagent and allowed to stand at 22&deg;C for 5 minutes before adding 0.75 ml sodium bicarbonate (60 g/L). The mixture was incubated at 22oC for 90 minutes and then read at 725 nm wavelength. Rutin was used as standard for the construction of calibration curve. The assay was carried out in triplicates.</div>\r\n<div>\r\n	2,2-diphenyl-1-picrylhydrazyl (DPPH) Assay</div>\r\n<div>\r\n	The radical scavenging activity of each extract was determined using 2,2-diphenyl-1-picrylhydrazyl (DPPH) assay as described byUllah et al, 2009 (Ullah et al. 2009). 0.25ml aliquots of each of the CA extracts or bioactive compounds (10-1000&mu;g/ml) were added to 0.75ml of freshly prepared ethanol DPPH solution (0.02mM). The optical density changes were measured at 517nm after 30 minutes. Blanks were used to remove the influence of colour of the samples. Ethanolic solution of DPPH was used as negative control. Butylated hydroxytoluene (BHT) and ascorbic acid were used as positive controls in the same concentration range (50-1000 &mu;g/ml) as were used for the samples. Free radical scavenging activity was expressed as inhibition percentage and was calculated using the following formula :</div>\r\n<div>\r\n	Ferric Reducing Antioxidant Power (FRAP) Assay</div>\r\n<div>\r\n	Working FRAP reagent was prepared by mixing 25 ml of acetate buffer pH 3.6, 25 ml of TPTZ (2,4,6-tripyridyl-s-triazine) solution, 2.5 ml of FeCl3.6H20 solution and 3 ml of distilled water (Benzie &amp; Strain 1996). The reaction of mixture of aliqout of CA extracts and bioactive compounds (25&mu;l) with working FRAP reagent (175&mu;l) was monitored for up to 4 minutes and the optical density changes were read at 593 nm. FRAP values were obtained by comparing the absorbance change at 593 nm in test reaction mixtures with those containing ferrous ion in known concentration using FeSO4.7H2O solution.</div>\r\n<div>\r\n	COX Enzymes Assay</div>\r\n<div>\r\n	Anti-inflammatory activity of CA extracts and bioactive compounds was evaluated by its ability to inhibit the inflammatory pathway enzyme, cyclo-oxygenase (COX) which catalyses the production of PGE2. This was determined using COX Inhibitor Screening Assay Kit ( Cayman Chemical ACE, USA). 20&mu;l of extract solutions (30&mu;g/ml) or bioactive compounds (5&mu;g/ml) were added to the COX-1 and COX-2 tubes respectively. The mixtures were incubated for 10 minutes at 37oC. The reaction was initiated by the addition of 10&mu;l of arachidonic acid to all the test tubes. The tubes were shaken vigorously and allowed to stand for another 2 minutes at 37oC. After that, 50&mu;l of 1 M HCl was added to each test tube to stop the reaction. Test tubes were removed from the water bath and 100&mu;l of the saturated stannous chloride solution was added to each test tube. The tubes were then shaken vigorously before leaving them to stand for 5 minutes at room temperature. The prostaglandin E2 (PGE2) produced were then quantified using a commercial Prostaglandin E2 Monoclonal-EIA Kit ( Cayman Chemical ACE, USA).</div>\r\n<div>\r\n	Suppression of TPA-Induced Prostaglandin E2 Production</div>\r\n<div>\r\n	The ability of the extracts, asiaticoside and madecassoside to suppress TPA-induced inflammation was evaluated by measuring the production of PGE2 which is produced during the inflammatory process. Fibroblast cells were incubated with TPA and either of the extracts, asiaticosside or madecassoside for 24 hours. The amount of PGE2 produced was measured using the Prostaglandin E2 Monoclonal-EIA Kit (Cayman Chemical ACE, USA).</div>\r\n<div>\r\n	Cells (5 x 103) were seeded in Dulbecco modified Eagle&rsquo;s medium (DMEM) supplemented with 10% fetal calf serum (FCS) in a 96-well plate and were incubated in a humidified atmosphere with 5% CO2 at 37oC, for 24 hours. Cells were then incubated with 12-O-tetradecanoylphorbol-13-acetate (TPA) and either of the extracts, asiaticosside and madecassoside for a further 24 hours. The supernatant from the tissue culture was then collected and quantified by EIA procedure.</div>\r\n<div>\r\n	RESULTS AND DISCUSSIONTotal Phenolic and HPLC Analysis</div>\r\n<div>\r\n	The level of total phenolic constituents, asiaticoside and madecassoside in the three different extracts are shown in Table 1.</div>\r\n<div>\r\n	The results showed that ethanol is the most effective solvent in extracting phenolic compounds as well as the bioactive compounds, asiaticoside and madecassoside. The total concentration of phenolic compounds extracted in this study for the three different extraction solvents was higher than that previously reported by Zainol et al. (2003). The levels of asiaticoside and madecassoside in both ethanol and methanol extracts of CA were in agreement with previous study which reported that values of triterpenic acid sugar esters including asiaticoside and madecassoside of 1-8% of plant material (Kartnig &amp; Hoffmann-Bohm 1992) and higher than that reported by Jayathirta &amp; Mishra (2004), who reported a yield of only 0.18 % of asiaticoside.</div>\r\n<div>\r\n	However the yield of asiaticoside (0.28%) using aqueous extraction produced lower yield (4%) than that reported by Guo et al. (2004). The low yield of asiaticoside and madecassoside in our aqueous extract could be due to the use of high temperature during extraction of plant materials. It has been reported that, high temperatures would decrease the concentration of asiaticoside, and madecassoside (Kormin 2005). Distribution of asiaticoside and madecassoside in C. asiatica plant were also organ specific, with the highest concentration found in leaves (Aziz et al. 2007). Plant origin could also be a factor for variations in asiaticoside content, as Das &amp; Mallick (1991) has reported that C. asiatica growing at higher altitude contained more asiaticoside than plants from lower altitude.</div>\r\n<div>\r\n	Antioxidant Activity by DPPH and FRAP Assay</div>\r\n<div>\r\n	The 2,2-diphenyl-1-picrylhydrazyl (DPPH) assay was used to determine the antioxidant activity of CA extracts based on their ability to trap unpaired electrons of DPPH. Extracts and compounds that could decolorize DPPH solution (purple) to yellow suggest their antioxidant potential. The DPPH scavenging activity of CA extracts in the concentration range of 50 - 1000 &mu;g/ml are shown in Figure 1.</div>\r\n<div>\r\n	Among CA extracts, the aqueous extract showed the strongest antioxidant activity at 1000 &mu;g/ml reaching a 79.4 % of DPPH radicals scavenging activity compared to methanol extract and ethanol extract with 77.3 % and 65.7 % respectively, at the same concentration level. However, both asiaticoside and madecassoside did not show any detectable DPPH scavenging activity, hence data is not shown.</div>\r\n<div>\r\n	The ferric-reduction ability of plasma (FRAP) assay also enables the evaluation of antioxidant potential based on the ability to reduce ferric ion (Fe3+) to ferrous ion (Fe2+). The antioxidant potential is shown as FRAP reagent decolorizes from purple to blue, as a result of the formation of ferrous-tripyridyltriazine (TPTZ) complex. The FRAP values of CA extracts in the concentration range of 500 &mu;g/ml - 2000 &mu;g/ml are shown in Table 2. At all concentrations, all CA extracts reduced the ferric-diTPTZ less than ascorbic acid and tocotrienol-rich fraction (TRF) that were used as positive controls. Among the extracts, aqueous extract showed slightly better ferric reducing ability than ethanol and methanol extracts at all concentrations tested. Consistent with DPPH assay, both asiaticoside and madecassoside did not show appreciable ferric reducing activity, hence data not shown.</div>\r\n<div>\r\n	Despite having the lowest total phenolic content among the CA extracts, aqueous extract possessed the strongest free radical scavenging activity as determined by the DPPH assay. Based on a previous study, the antioxidant activity of polar extracts could be due to the presence of compounds with free hydroxyls, such as flavonoids (Mensor et al. 2001). Levels of flavonoids in the aqueous extract of CA could be higher compared to ethanol and methanol extract due to it being extracted in most polar water, compared to less polar ethanol and methanol solvent. Flavonoids make good antioxidant agents since they possess a number of free hydroxyls which can scavenge free radicals (Cao et al. 1997; Basile et al. 2005). In contrast to our findings, a previous study on CA extraction has shown that ethanol extract had the highest antioxidant activity when compared with aqueous extract (Hamid et al. 2002). However, the differences might be due to different methods of extraction and assays used to measure the antioxidant activity. The results also suggest that the antioxidant activity of CA resides in water soluble compounds. This is also in agreement with several reports that high polarity solvents are effective in extracting natural antioxidants (Duh et al. 1992; Tian &amp; White 1994).</div>\r\n<div>\r\n	Anti-Inflammatory Activity</div>\r\n<div>\r\n	The concentration of CA extracts (30 g/ml) and the two bioactives (5 &mu;g/ml) used for the anti-inflammatory studies were predetermined using MTS assay to ensure cell viability (data not shown).</div>\r\n<div>\r\n	The CA extracts and bioactive compounds were tested for COX-1 and COX-2 enzyme inhibitory activity. At 30 &mu;g/ml, the ethanol, methanol and aqueous extracts inhibited COX-1 and COX-2 enzymes by 97.84% and 97.91%, 98.18% and 96.16%, 83.06% and 72.10%, respectively. Asiaticoside and madecassoside at 5 g/ml concentration inhibited COX-1 and COX-2 enzymes by 98.08% and 83.67%, and 91.57% and 89.79%, respectively. Aspirin was used as positive control at 5 mg/ml concentration and inhibited COX-1 and COX-2 by 80.98% and 88.86%, respectively (Fig.2). Among CA extracts, ethanol and methanol extracts showed stronger inhibition of COX-1 and -2 than the aqueous extract. Asiaticoside and madecassoside inhibitory activity of COX-1 and COX-2 enzymes were similar to commercial NSAIDs, aspirin.</div>\r\n<div>\r\n	The anti-inflammatory activity of CA extracts and compounds were further studied using TPA-induced inflammation in human fibroblasts. Treatments with 30 g/ml concentration of ethanol, methanol and aqueous extracts of CA on the TPA-induced fibroblast cells produced significantly less PGE2 compared to control (TPA alone). Treatment of the cells with asiaticoside and madecassoside at 5 g/ml also significantly reduced the TPA-induced formation of PGE2 (Figure 3).Figure 3 also showed that ethanol and methanol extracts of CA were more potent than aqueous extract in suppressing the TPA-induced formation of PGE2 while Figure 2 showed that these extracts were better inhibitors of the COX activity.</div>\r\n<div>\r\n	Bioactive compounds of CA, asiaticoside and madecassoside suppressed PGE2 formation at similar level. Both compounds were also found to inhibit TPA-induced inflammation in fibroblasts by modulating COX activity as shown by their inhibitory effects in COX-1 and -2 enzymes assay. Our findings also agree with previous work on collagen-induced arthritis mice in which treatment with asiaticoside were reported to inhibit inflammation by reducing the expression of COX-2 and inflammatory cytokines (Li et al. 2007). Previous work on similar model of arthritis, have found that oral administration of madecassoside to reduce the expression of COX-2 and PGE2 production, but did not change the expression of COX-1 (Li et al. 2009).</div>\r\n<div>\r\n	The more potent anti-inflammatory activity of ethanol and methanol extracts of CA in regressing inflammation in TPA-induced fibroblasts than the aqueous extract could be due to higher content of phenolics, and or bioactive compounds asiaticoside and madecassoside in both ethanol and methanol CA extracts compared to the aqueous extract. Asiaticoside and madecassoside, both are shown to have anti-inflammatory activity, and these compounds may react synergistically with each other or with other phenolic constituents to give CA its anti-inflammatory activity.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The present study demonstrated that hydrophobic solvent extraction of Centella asiatica yielded better yields in terms of phenolic, asiaticosside and madecassoside contents and that this is associated with better anti-inflammatory potential.</div>\r\n<div>\r\n	ACKNOWLEDGMENTS</div>\r\n<div>\r\n	The authors would like to thank the Ministry of Science, Technology and Environment of Malaysia for supporting the study (Grant No. 02-01-02-SF0525) and Medical Faculty, Universiti Kebangsaan Malaysia for the laboratory facilities.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1000, 994, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Acute leukaemia is the commonest cancer in children representing a third of all childhood malignancies (Greaves 2002). In Malaysia, the incidence of leukaemia is 9.5 per 100 000 population for males aged between 0 to 14 years, and 6.4 per 100 000 population for females aged between 0 to 14 years (Lim &amp; Halimah 2000; Ministry of Health 2000). Currently, the use of multi-drug regimens and central nervous system (CNS) prophylaxis is largely responsible for the overall 85% chance of long term survival in this group of children (Pui 2000).</div>\r\n<div>\r\n	The life-threatening nature of acute leukaemia and its invasive treatment present both physical and emotional stress for parents. Parenting stress is defined as total stress that a parent experiences as a function of certain salient child characteristics, parent characteristics and situations, which are directly related to the role of being a parent (Hoekstra-Weebers et al. 1998). High levels of parenting stress have been found in many studies during the time of diagnosis and throughout the early stage of treatment, which persist for more than a year (Sawyer et al. 1993; Kazak et al. 1995). However, in a 4-year prospective study of psychological adjustment of parents whose children were diagnosed with cancer, Sawyer et al. (2000) found no significant differences between the psychological problems reported by parents whose children had been diagnosed with cancer and a control group whose children were healthy (Sawyer et al. 2000).</div>\r\n<div>\r\n	Failure to cope and to deal adaptively with stress may result in mental and physical illness. Coping has been defined as &ldquo;constantly changing cognitive and behavioural efforts to manage specific external and/or internal demands that are appraised as taxing or exceeding the resources of the person&rdquo; (Pearlin &amp; Schooler 1978; Lazarus &amp; Folkman 1984). In general, people who rely more on approach-oriented and task-oriented coping mechanisms tend to adapt better to life stressors and experience fewer psychological problems (Folkman &amp; Lazarus 1988). Conversely, avoidance and emotion-oriented coping strategies seem to be associated with psychological distress. These latter two strategies involve emotional responses, and their behaviour and thoughts tend to focus on depressive symptoms (Ray et al. 1982).</div>\r\n<div>\r\n	Attention to the psychosocial needs of families during cancer treatment, particularly with respect to parental anxiety, may help these families cope with the treatment process and prevent them from dropping out from cancer treatment for childhood cancer. The purpose of this study was (i) to determine the level of parenting stress amongst parents of children who have been diagnosed with acute leukaemia; (ii) to determine risk factors associated with high parenting stress score and (iii) to determine the coping mechanism used by parents in relation to stress.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a single centre, cross-sectional study conducted in UKM Medical Centre. The study was conducted from August 2007 till July 2009. The patients and their parents were recruited from the paediatric haematology/oncology clinic, ward and day care centre. The sampling method was universal sampling. All children between the age of one month to 12 years old, diagnosed to have acute leukaemia (acute lymphoblastic leukaemia, ALL or acute myeloblastic leukaemia, AML) were included. These patients were either still receiving or had completed chemotherapy. Children with other co-existing chronic illnesses or secondary malignancies and non-Malaysian citizens were excluded from the study. For the purpose of this study, a child who is newly diagnosed is defined as a child on treatment within the first two months following diagnosis. This would include those on induction and consolidation phase of chemotherapy.</div>\r\n<div>\r\n	Written informed consent was obtained from the parents after explanation regarding the study was carried out. The questionnaires were distributed to the parent or both parents, depending on their presence during the recruitment process; if both parents were present, the questionnaires were answered independently. The following questionnaires were distributed to each parent individually: Parenting Stress Index/Short Form (PSI/SF) and Coping Inventory for Stressful Situation (CISS).Both questionnaires were available in Bahasa Malaysia or English language and have been validated (Farah Inaz 2004; Salwina 2001). These self&ndash;administered questionnaires were collected immediately upon completion. If the parents were not able to complete the questionnaires immediately, pre-stamped envelopes were provided. If the forms were still not received within the next two weeks, a reminder telephone call was made.</div>\r\n<div>\r\n	The 36-item PSI/SF is a direct derivative of the full-length (120 item) PSI through a series of replicated factor analyses (Abidin 1995). The PSI/SF comprises three subscales: parental distress (PD), parent-children dysfunctional interaction (PCDI) and difficult child (DC). The PD subscale measures the distress directly related to parenting a sick child. The PCDI subscale focuses on the parents&rsquo; perceptions that his/her child does not meet the parents&rsquo; expectations and that the interactions with his/her child are not reinforcing to his/her parent. Whereas the DC subscale represents basic behavioural characteristics of children that parents believe make them either easy or difficult to handle. Each of these subscales contains 12 items; the total score of the PSI/SF is obtained by adding the three subscales together; a 5 point Likert scale is used, ie from &lsquo;strongly agree&rsquo; to &lsquo;strongly disagree&rsquo;. The maximum score for each subscale is 60 and the maximum score for the entire PSI/SF is therefore 180; the higher the score, the higher the parenting stress. The total score for PSI is divided into centile range. In our study, high parenting stress score is defined as a total PSI score of more or equal to the 75th centile.</div>\r\n<div>\r\n	The CISS was designed to measure the mechanism of coping. It has a total of 48 items with statements on a five point Likert scale ranging from &lsquo;never&rsquo; to &lsquo;very often&rsquo;. The items are subdivided into three domains: emotion, avoidance, and task-oriented (Endler &amp; Parker 1990). Task-oriented coping describes purposeful task-oriented efforts aimed at solving the problem and cognitively restructuring the problem; this includes attempts in seeking information and attempts to alter the situation. Emotion-oriented coping describes self-oriented emotional reactions which may actually increase the level of stress; these reactions include becoming emotional or feeling angry and tense in response to the situation. Avoidance-oriented coping describes activities and cognitive changes aimed at avoiding the stressful situation. Examples of this include denial, wishful thinking and withdrawal.</div>\r\n<div>\r\n	All data were analysed using the SPSS version 16.0. Chi square test was used for comparison of qualitative variables between the high parenting stress group and low parenting stress group. Unpaired t test was used for parametric analysis of risk factors that affect parenting stress. Mann Whitney U and Kruskal Wallis tests were used for non-parametric bivariate analysisANOVA was used for parametric bivariate analysis comparing two or more population means such as mean PSI and DC score in the various treatment phase. Post Hoc analysis was used to re-analyse risk factors that were found to be significant based on ANOVA. Logistic regression analysis was used to identify factors which affected parenting stress the most.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	There were 106 children with acute leukaemia who were on follow up at the Paediatric Haematology and Oncology Unit of UKMMC during the study duration. Eighty nine patients fulfilled the inclusion criteria. Seventeen patients did not meet the inclusion criteria and were excluded from the study: of these, 13 were beyond 12 years of age; two had parents who were not able to read nor write in both languages; one had secondary leukaemia and another was a non-citizen. From the 89 patients who fulfilled the inclusion criteria, 16 were missed during the recruitment process due to either infrequent clinic appointment or defaulted follow up. From the remaining 73 children who were eligible for the study, only 66 families responded to the questionnaires which comprised 56 fathers and 61 mothers.</div>\r\n<div>\r\n	The mean age of patients was 7.9&plusmn;2.8, years and slightly more males (56.1%). The majority were of Malay ethnicity (75.8%). Thirty patients (45.4%) were on maintenance chemotherapy and 26 patients (39.4%) had completed treatment. Eight patients (12.1%) were newly diagnosed and two were on palliative treatment as they were not in remission despite receiving treatment. Table 1 summarises the socio-demographic characteristics of the parents. The majority (75.2%) had three or more children. Most of the parents (66.6%) received education till secondary school level. A total of 73 parents (62.4%) made mutual decision in relation to their child&rsquo;s treatment. The mean total parenting stress index (PSI) score was 91.5&plusmn;21.1 (Table 2). The mean score for coping mechanism was highest for the task-oriented coping mechanism i.e. 59. Statistical analysis showed that there was no significant difference between the mean maternal and paternal PSI [i.e. 91.3 (&plusmn;22.7) and 91.6 (&plusmn;19.4) respectively], and CISS scores. Table 3 illustrates that there was no significant difference in the mean score for each of the PSI/SF subscales based on the various phase of treatment for acute leukaemia. Two parents whose children were on palliative care were omitted from this analysis.</div>\r\n<div>\r\n	Risk factors for high parenting stress scores were categorised into parental or patient-related factors. Parental risk factors associated with high parenting stress scores (&ge; 75th centile which correspond to a total score of &ge; 103) are shown in Table 4. Emotion-oriented coping mechanism was significantly associated with a high parenting stress score (p value 0.009); none of the other factors were statistically significant. There was no significant difference in total parenting stress score between parents who made mutual decisions as compared to those who did not (p value 0.9).</div>\r\n<div>\r\n	As for patient-related risk factors, none were statistically significant; these included age, gender, race, religion and treatment phase. Multiple logistic regression analysis was performed to predict the probability of having a high total PSI score (&ge; 75th centile). Emotion-oriented coping mechanism was found to be significant with a p value of 0.03. A parent who uses emotion-oriented coping mechanism was 7.1 (95% Confidence Interval 1.2 to 41.4) times more likely to have a high total PSI score as compared to a parent who uses other types of coping mechanisms.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The diagnosis and subsequent treatment of childhood leukaemia is undeniably stressful for any family. A parent&rsquo;s ability to manage his or her distress during treatment of the child is vital as there may be potential impact on the well-being and long-term psychological adjustment of both parents and child.</div>\r\n<div>\r\n	The mean total PSI score in our study was 91.50 (&plusmn;21.12) which was lower when compared to the mean total PSI score amongst Malaysian mothers of children with epilepsy [97.8 (&plusmn;20.99)] (Farah Inaz 2004) and spina bifida [92.4 (&plusmn;11.43)] (Norshireen Nazli 2005). In these two local studies, the control groups had a mean total PSI score of 85.07 (&plusmn;18.77) and 78.2 (&plusmn;21.14) respectively. Epilepsy and spina bifida are diseases in which treatment is largely symptomatic and non-curative. Spina bifida is also usually associated with physical disabilities in comparison to leukaemia; this may have contributed to a higher score within the PD and DC subscales of the PSI/SF.</div>\r\n<div>\r\n	In a study by Yeh amongst Taiwanese parents of children with cancer, the mean maternal and paternal scores were 124.1 (&plusmn;19.47) and 125.86 (&plusmn;18.64) respectively; there was no statistical difference based on parental gender (Yeh 2002). Although our study also revealed no significant difference between the gender of parents, the mean total PSI scores for their parental groups were much higher when compared to our results. Cross-cultural differences may have contributed to the higher parenting score in the Taiwanese population. The non-significant difference between maternal and paternal parenting scores probably infers that the overall responsibility and care of the child were shared equally by both parents. This is also reflected by the fact that decisions were mutually made by the majority of parents (62.4%) in our study. However, the non-significant difference between maternal and paternal parenting stress scores could also be explained by the possibility of both parents discussing the questions (of the research tools) and sharing their responses.</div>\r\n<div>\r\n	Only 27.3% of the parents from our study had a high total parenting stress score. Published studies have reported a variable prevalence of sub-clinical parenting stress related symptoms amongst parents caring for children with leukaemia which ranged from 20% to 95% (Kazak et al. 2001; Kazak et al. 2004; Manne et al. 1998). This was however based on other forms of assessment tools in determining parenting stress level. High level of parenting stress has been found in many studies both at the time of diagnosis and early stage of treatment which may persist for more than a year (Hoekstra-Weebers et al. 2001). Our study has shown that there was no difference in the total PSI score throughout the various phase of treatment for leukaemia.</div>\r\n<div>\r\n	Several studies have reported a higher parenting stress level amongst parents with lower income group and lower educational level (Young et al. 2002). Our study however showed no significant difference in both these factors. The cost of chemotherapeutic treatment in our patients is significantly supported by our hospital in addition to financial support by various non-governmental organisations. The majority (66.6%) of parents in our study have an educational level of secondary school; this may have also contributed to better parental understanding, perception and acceptance of the child&rsquo;s disease and treatment.</div>\r\n<div>\r\n	The style or mechanism of coping plays an important role in individual well-being. In our study, task-oriented coping mechanism was used by the majority of parents. As mentioned before, task-oriented coping is a strategy involving problem solving and seeking information in order to alter the situation. This could probably explain why only 27.3% of parents in our study had a high total PSI score (&ge; 75th centile). Conversely, avoidance and emotion-oriented coping strategies seem to be associated with psychological distress. Interestingly, emotion-oriented coping mechanism was the only identifiable risk factor for high total PSI score following multiple logistic regression analysis in our study. Emotion-oriented coping mechanism is said to be bi-directional. In the short term, emotion-oriented coping may be beneficial as it allows the parent to ventilate and express their emotions more freely. In the long run however, it can be maladaptive as it may result in more stress. Thus, parents who use emotion-oriented coping strategy should be identified early so that psychosocial support may be given to alleviate the stress.</div>\r\n<div>\r\n	There were several limitations to our study. As this was a cross-sectional study, the collection of data was only done at a particular point in time. As a result, evaluation of parenting experiences over the entire course of illness or treatment was not possible. A longitudinal study with repeated assessment of parenting stress and coping mechanisms at various point in time would have been a better option. The use of disease-specific measures of parenting stress, such as Pediatric Inventory for Parents and the Psychosocial Assessment Tool may have been beneficial and thus provided more accurate levels of parenting stress within the context of childhood illness than the more general measures of parenting stress (Streisand et al. 2001; Kazak et al. 2003). In addition to this, life stressors were also not assessed concurrently. Parental personality traits prior to the onset of the child&rsquo;s illness is another important aspect in the assessment of coping strategy and it is preferable to evaluate these traits when dealing with coping measures for future studies. Therefore, a prospective study, incorporating additional assessment tools such as life stressor and child behavioural checklist, is recommended for better understanding of parental stress over the entire course of treatment.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The mean total parenting stress score amongst parents of children diagnosed with acute leukaemia was 91.5&plusmn;21.1 which was lower than that reported by other studies involving cancer patients. Only 27.3% of these parents experienced a high total parenting stress score. Emotion-oriented coping mechanism was the only risk factor that was associated with a high PSI score; as such the CISS may be used as a screening tool to identify these at-risk parents so that psychosocial intervention may be offered.</div>\r\n', 1),
(1006, 1000, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Once an unprotected skin is exposed to the sunlight, ultraviolet (UV) radiation which is absorbed by the skin chromophores interacts with oxygen molecules to generate reactive oxygen species (ROS) such as hydrogen peroxide, superoxide radical anion, hydroxyl radical and singlet oxygen. Eventually, these hazardous ROS damages the neighbouring deoxyribonucleic acid (DNA), proteins, and lipids in the skin (Bastuji-Garin et al. 2002). Therefore, prolonged and repeated exposures to sunlight would cause skin problems such as photodamage, photoaging and skin cancer (Gilchrest 2008).</div>\r\n<div>\r\n	Sunscreen is claimed to be the gold standard in protecting our skin from photodamage as it contains UV filters that may be present in the form of organic compounds, inorganic molecules or organic particulates (Seite 2000). When the skin is exposed to UV light, UV filters in sunscreen absorb, reflect and scatter the UV irradiation thus preventing it from reaching the skin. Antioxidants have been used as an adjuvant to sunscreens because of their ability to neutralize ROS generated by UV radiation (Seite 2000). Hence, a vast number of sunscreens containing antioxidant additives have been introduced in the market.</div>\r\n<div>\r\n	Sun Protection Factor (SPF) which measures the amount of protection against UVB (290-320 nm), is a multiple of UV irradiance required to cause erythema on a sunscreen applied skin as compared to a bare skin. A high SPF value indicates a better protection from sun burning of the skin. On the other hand, Ultraviolet A protection factor (UVAPF) measures the amount of protection to UVA (320-400 nm) radiation afforded by sunscreens where a high value indicate a better protection from persistent pigment darkening of the skin when exposed to sunlight (Diffey et al. 2000). In our search for sunscreen samples for this study, most sunscreens were found to have no information regarding this value.</div>\r\n<div>\r\n	Photostability is defined as the ability of a product to retain its integrity upon exposure to light. A product is considered photostable when the reduction of SPF and UVAPF values are less than 20% after exposure to light (Garoli et al. 2008). A research conducted by Herzog and Sommer (2000) found that chemical compounds in sunscreens that are unstable with exposure to sunlight may serve as another source of free radicals upon degradation.</div>\r\n<div>\r\n	Steven et al. (2010) found that antioxidant additives in some sunscreens had no effect in naturalizing ROS developed in photodamaged skin. Various reasons were postulated as the cause of this finding such as low antioxidant power, low biological activity, insufficient concentration of antioxidant as well as the inability of antioxidant to penetrate the skin. Thus, more effective methods and means of adding antioxidants to improve photoprotection are much needed.</div>\r\n<div>\r\n	Therefore, the aim of this study is to compare the photoprotective measures of some commercially available sunscreens in Malaysia. The efficacy and safety of international and local branded sunscreens is compared in terms of their SPF label accuracy, relative UVAPF level and their photostability. The adjuvant effects of adding concentrated vitamin C and E creams in improving photoprotection is also assessed since these vitamins have been tested to protect against skin cancer and photoaging though their efficacy towards SPF and UVAPF values have yet to be determined to date (Lin et al. 2003).</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Sample Size</div>\r\n<div>\r\n	The sample size was calculated based on Mead&rsquo;s Resource Equations 1988. The error of degree of freedom was set in between 10-20. A total of three local and three international manufactured sunscreens were analysed in triplicates in this study, based on the European Cosmetic Products Trade Association (COLIPA) guideline for UVAPF 2009 determination (Table 1).</div>\r\n<div>\r\n	Operational Definitions</div>\r\n<div>\r\n	The international and local sunscreens were selected from a range of commercially available sunscreens that were manufactured outside and in Malaysia respectively.</div>\r\n<div>\r\n	Inclusion Criteria</div>\r\n<div>\r\n	The selected sunscreens contained no antioxidants and had details of the country from where they were manufactured. The antioxidant creams contained mainly vitamins C or E as the active ingredient. Table 2 shows the details of sunscreens and antioxidant creams used in the experiment.</div>\r\n<div>\r\n	Method</div>\r\n<div>\r\n	Roughened polymethylmethacrylate (PMMA) plates with measurement of 40mm x 40mm x 2.5mm from NIC Advertising, Ampang, Selangor, Malaysia were used. An amount of 2mg/cm2 of sunscreens alone and 4mg/cm2 of the combinations of sunscreens with Vitamin C or/and E creams was added onto the plate with the aid of a weighing scale with variations 5% of weight. The test samples were applied onto the plate as multiple small droplets and then spread as even as possible. The test sample was then allowed to stabilize for 15 minutes in a dark, air conditioned room.</div>\r\n<div>\r\n	The absorbance profile of the test samples were measured by a two-beam UV/VIS Spectrophotometer Shimadzu UV-2450 spectrophotometer.</div>\r\n<div>\r\n	Polymethylmethacrylate (PMMA) plate covered with test samples was placed in a plate holder of the spectrophotometer. The UV absorbance of each test sample was measured from 290-400 nm at 1 nm intervals at five different places per plate. The data was read using UVProbe 2.33. The average of five readings was taken for analysis.</div>\r\n<div>\r\n	Since consumers are commonly advised to reapply sunscreens every two hours (Diffey 2001), the test samples were also exposed under the sun for two hours (12:00 pm-14:00 pm). Table 3 shows the UV index of the day of the experiment provided by the Malaysian Meteorological Department, Petaling Jaya, Selangor, Malaysia. Hence, the average UV index for two hours of irradiation was 4.74.</div>\r\n<div>\r\n	The absorbance profile of the test samples after irradiation was taken again. All data collected was analysed using Statistical Product and Service Solutions (SPSS) version.20.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Figure 1 shows examples of absorbance profiles of test samples. The shape and height of the curves varied depending on the type and concentration of UV filters used in the test samples. The in vitro SPF and UVAPF values as well as photostability were calculated based on the listed formulas.</div>\r\n<div>\r\n	Local vs International Sunscreens</div>\r\n<div>\r\n	SPF Label Accuracy</div>\r\n<div>\r\n	Coefficient of adjustment (C) was used as a standard to measure the accuracy of SPF label of the sunscreen. It was calculated by using formula 4. Values between 0.8-1.2 meant the SPF label was accurate. Figure 2 shows SPF label and in vitro SPF of various sunscreens.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A total of nine (100%) international sample plates had accurate SPF label while only three (33.3%) local sample plates had C values that fell between 0.8-1.2. A Fisher&rsquo;s exact test was computed to test the association between manufacturing places and the SPF label accuracy. The result revealed that there was significant association between the manufacturing place and the SPF label accuracy. The international sunscreens had more accurate SPF label than local sunscreens (p=0.009). The details of the analysis are shown in Table 4.</div>\r\n<div>\r\n	Relative UVAPF Level</div>\r\n<div>\r\n	According to the European Commission 2006, sunscreens should provide UVAPF of at least one third of the SPF label to ensure balanced and adequate protection against both UVA and UVB radiation. Figure 3 shows the comparison of relative UVAPF level between local and international sunscreens. There were only two (22.2%) international sunscreens with acceptable UVAPF to SPF label ratio. Fisher&rsquo;s exact test proved that most of the sunscreens regardless of the manufacturing place were not providing enough UVA protection (p=0.471) as shown in Table 5.</div>\r\n<div>\r\n	Photostability</div>\r\n<div>\r\n	Figure 4 displays the photostability of international and local sunscreens. Local sunscreens scored a mean rank of 13.22 while the mean rank for international sunscreens was 5.78 only in Mann-Whitney U test (Table 6). Hence, local sunscreens were more photostable than international sunscreens (p=0.003).</div>\r\n<div>\r\n	Roles of Antioxidant Creams in Photoprotection</div>\r\n<div>\r\n	Differences Before and After Adding Antioxidants onto Sunscreens</div>\r\n<div>\r\n	A comparison between in vitro SPF, in vitro UVAPF and photostability of sunscreens alone, sunscreens added with Vitamin C cream, sunscreens added with Vitamin E cream and sunscreens added with a combination of Vitamins C and E cream was performed. Table 7 shows the differences before and after adding antioxidants into sunscreens according to the above-mentioned combination. With reference to the Kruskal-Wallis tests, addition of either Vitamin C or E creams with sunscreens improved the in vitro SPF (p=0.009), in vitro UVAPF (p=0.007), and photostability (p=0.002).</div>\r\n<div>\r\n	Differences between the Photoprotection Levels of Vitamins C and E Cream</div>\r\n<div>\r\n	Vitamin C cream enhanced the SPF of the sunscreens (p=0.040) while Vitamin E made the sunscreens more photostable (p=0.000). However, there is no significant differences between Vitamin C and E in improving UVAPF (p=0.054) of the sunscreens (Table 8).</div>\r\n<div>\r\n	The Synergistic Effects of Combining both Vitamins C and E Cream</div>\r\n<div>\r\n	We observed that combining both Vitamins C and E cream into a sunscreen did not significantly improve the SPF and UVAPF (p&gt;0.005) compared to the use of a single antioxidant cream. More importantly, the above combination also showed lower photostability (p=0.002) compared to adding Vitamin E alone (Table 9).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Local vs International Sunscreens</div>\r\n<div>\r\n	SPF Label Accuracy</div>\r\n<div>\r\n	Results of analysis showed that local sunscreens had less accurate SPF label than international sunscreens probably because the selected locally produced sunscreens did not comply with the Good Manufacturing Practice (GMP) guidelines which recommend products to be tested in terms of quality, packaging, safety, toxicology and labeling before being awarded with the certificate.</div>\r\n<div>\r\n	Relative UVAPF Level</div>\r\n<div>\r\n	Our result is consistent with the finding from Environmental Work Group (EWG) 6th Annual Sunscreen Guide 2012 where most of the sunscreens were found to provide inadequate UVA protection. However information regarding UVAPF provided by the sunscreens was not a requirement in passing the Safety Assessment of Cosmetic Product in any country. Most sunscreens were found to screen out UVB only. Thus, prolonged exposure to sunlight does increase the risk of melanoma despite the use of sunscreens (Gorham 2007).</div>\r\n<div>\r\n	Photostability</div>\r\n<div>\r\n	After two hours of irradiation under the sun, all sunscreen samples had decreased in vitro SPF values. This might be due to degradation of UV filters with exposure to irradiation which decreases the photostability of sunscreens thus leading to lower SPF values.</div>\r\n<div>\r\n	According to the EWG Skin Deep Sunscreen Guideline 2011, low-SPF products contained fewer amounts of UV filters than high-SPF sunscreens. Consumers are not recommended to use sunscreens with high SPF values because degradation of excessive UV filters during exposure to sunlight produces ROS which damages the skin (XX). Sunscreens with high SPF values are thus suitable only for short duration of sun exposure. In our study, SPF values for locally produced sunscreens were 50% lower than the international sunscreens. Hence, locally produced sunscreens are suggested to be more photostable than international sunscreens.</div>\r\n<div>\r\n	Roles of Antioxidant Creams in Photoprotection</div>\r\n<div>\r\n	Using vitamin C, vitamin E or the combination of both creams together with sunscreens are found to improve the function of sunscreens in terms of SPF, UVAPF and photostability.</div>\r\n<div>\r\n	According to Dominique Lutz et al. (1999), SPF and UVAPF values are affected by the concentration of UV filters and the thickness of sunscreens applied. This is in line with our study which found that the increased thickness of applied sunscreens upon application of antioxidant creams showed increased values of SPF and UVAPF. In other words, antioxidant creams are able to enhance the photoprotection of sunscreens without increasing the concentration of UV filters.</div>\r\n<div>\r\n	In addition, photostability of the sunscreens is also improved with the use of antioxidant creams. This may be due to the capability of antioxidants to neutralize free radicals generated from UV light and degraded UV filters (Edlich et al. 2004). This in turn prevents subsequent generation of ROS which disrupts the integrity of sunscreen leading to increased photostability.</div>\r\n<div>\r\n	Vitamin C cream increased the in vitro SPF level more than vitamin E cream. Both vitamins C and E have been shown to have no UVB filtering functions (John et al. 2008). The increase in SPF value could have been contributed by minor additives in the Vitamin C cream such as Ethylhexyl Methoxycinnamate and Butyl Methoxydibenzolmethane which are UVB filters.</div>\r\n<div>\r\n	There is no significant difference between vitamins C and E in improving UVAPF of the sunscreens. As mentioned before, antioxidant creams act as a supplement to the sunscreen since they have no UV filtering properties.</div>\r\n<div>\r\n	Meanwhile, vitamin E cream showed higher efficacy in improving the photostability of the sunscreen compared to vitamin C. &alpha;-Tocopherol acetate (Vitamin E) is stable and will not be converted to its unstable free form &alpha;&alpha;-tocopherol if applied topically (Alberts et al. 1996). On the other hand, L- ascorbic acid (Vitamin C) will always be metabolized to its unstable counterpart by the skin (Sheldon et al. 2001). Though effort had been put in esterifying L-ascorbic acid to make it more stable in topical cream, unfortunately it is still less stable than &alpha;-tocopherol.</div>\r\n<div>\r\n	The Synergistic Effects of Combining Both Vitamins C and E Cream</div>\r\n<div>\r\n	The synergistic effects of combining both vitamins C and E cream were not observed in our experiment. This combination improved the SPF and UVAPF values insignificantly compared to adding Vitamin C or E alone. This is contradictory to findings by other researchers. The explanation for this insignificance is probably due to the same thickness that we applied for the three combinations.</div>\r\n<div>\r\n	Combining both Vitamin C and E onto sunscreens demonstrated lower photostability in comparison to adding Vitamin E alone. We postulated that portion of degraded Vitamin C reduces the stability of this combination.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The use of sunscreen alone is not effective regardless of the place of manufacture. Concentrated antioxidant creams should be used as adjuvant for photoprotection. As concurrent use of vitamins C and E does not improve the quality of sunscreen, it is best to apply a layer of sunscreen together with vitamin C rich cream for intense sun exposure or Vitamin E rich cream for longer duration of sun exposure.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	&bull; Department of Biochemistry and Physiology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Aziz, 50300 Kuala Lumpur, Malaysia</div>\r\n<div>\r\n	&bull; Malaysian Meteorological Department, Petaling Jaya, Selangor, Malaysia</div>\r\n<div>\r\n	&bull; This research was approved by the UKM Research Ethics committee and funded by Dana Fundamental UKM (FF-100-2012)</div>\r\n', 1),
(1011, 1005, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Tuberculosis is on the rise due to influx of foreign workers into the country. Ocular tuberculosis can either be immunogenic or infective in origin, however, the definitive diagnosis is challenging (Nissapatorn et al. 2007). Broadly, the clinical features of ocular tuberculosis give certain clues towards the underlying aetiology, supplemented by other tests. We report an interesting case of seemingly infective ocular tuberculosis in an immunocompetent patient, yet without any signs of inflammation. This case highlights the ocular features, the importance of early management and the difficulties in diagnosing the underlying aetiology of ocular tuberculosis.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 17-year-old male student of Indonesian parentage presented with two weeks history of painless and progressive visual deterioration without photopsia or floaters. He denied any contact with tuberculosis patients or any high- risk behavior. There was no reported back or joint pain, fever or rashes, nor any constitutional symptoms. According to his parents, all immunisation was up to date, including for Bacilli Calmette Gue`rin (BCG).</div>\r\n<div>\r\n	Visual acuity at presentation was 6/36, N48 and 6/60, N-for the right and left eyes respectively. There was no relative afferent pupillary defect, and both the anterior and vitreous chambers were quiet. On funduscopic examination, the right optic disc is swollen with macular star. There was vasculitis involving both arteries and veins with perivascular exudates and signs of branch vein occlusion bilaterally (Figure 1). In addition, occlusive vasculitis was seen on the left eye. Blotchy haemorrhages were seen with cotton wool spots and obvious macular oedema, as evidenced on optical coherence tomography (OCT) (Figure 1). There was no evidence of choroiditis.</div>\r\n<div>\r\n	Fluorescein angiography study confirmed large areas of capillary fallout in both eyes and leaking new vessels, consistent with ischaemia secondary to inflammation (Figure 2). Mantoux test was measured to be 15 mm and screening for Human Immunodeficiency Virus (HIV) was negative. Haematological parameters did not show any elevation of the white cell count or its differentials. Erythrocyte sedimentation rate showed increased level of 98 mmol/ hr. The VDRL test was negative and chest X- ray was clear. The patient&rsquo;s family declined to have quantiferron and polymerase chain reaction (PCR) tests done.</div>\r\n<div>\r\n	Based on the significant Mantoux test and elevated ESR level, he was diagnosed as ocular TB, although there was no sign of vitritis and anteriorchamber inflammation despite being immunocompetent. Full regime of anti- TB therapy was started constituting of rifampin 600 mg, ethambutol 800 mg, isoniazid 300 mg, and pyrazinamide 1000 mg once daily. His macula showed reduction in thickness on follow- up. He also received intensive panretinal laser photocoagulation. Three days following the institution of anti TB, patient was started on 1mg/ kg of oral steroid daily. New vessels showed signs of regression and macula oedema was resolving in both eyes. His visual acuity had improved to 6/12 and patient is currently still receiving treatment.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The incidence of tuberculosis is on the rise due to influx of foreign workers into the country and the increasing number of HIV positive patients (Dolin et al. 1994). Tuberculosis is a bacterial infection which does not only affect the lungs, but may also occur in any other organs, including the eye. Ocular tuberculosis may occur in isolation or as miliary infections from another source (Helm &amp; Holland 1993). The ocular manifestations of TB may either be immunogenic or infective in origin (Helm &amp; Holland 1993), however the definitive diagnosis between these two diagnoses may be challenging. The management of immunogenic ocular TB is different from the infective ones, hence the importance of correct diagnosis. Immunogenic TB requires treatment with immunomodulating agents such as steroid, whereas anti- TB medication is necessary for infective cases. However, the Jarisch- Herxheimer reactions may occur following initiation of anti- tuberculous therapy and may also require treatment with steroids (Neunh&ouml;ffer et al. 2013).</div>\r\n<div>\r\n	Our patient presented with occlusive retinal vasculitis without other signs of ocular inflammation, despite his immunocompetent status. Generally, immune- mediated and infective ocular TB is differentiated by its clinical features (Kurup &amp; Chan 2006), especially if laboratory results are indefinite. Immunogenic TB tends to present with vascular occlusive features associated with dense vitritis in immunocompetent patients (Thompson &amp; Albert 2005). Although infective ocular TB affecting the posterior segment usually manifests as multiple choroidal lesions, consistent with the haematologic spread of the bacteria (Sheu et al. 2001), no choroidal lesions were seen in this case. The QuantiFERON&reg;-TB Gold In-Tube test (QFT-GIT) is a valuable diagnostic tool to diagnose infective TB (Rutherford et al. 2010). However, this may not always be possible for each case due to financial limitation. Furthermore, this particular test does not differentiate between active or latent TB (Rutherford et al. 2010).</div>\r\n<div>\r\n	Tubeculin-skin (Mantoux) test measures the hypersensitivity reaction to tuberculin (Nayak &amp; Acharjya 2012). Although the test is neither 100% specific nor 100% sensitive, it is still widely used to screen against TB, especially in high risk populations. Combining both the Tuberculin- skin test and the gold Quantiferon assay may prove to yield better results in diagnosing TB (Rutherford et al. 2010). T-SPOT-TB is another available form of Interferon-Gamma Reactive Assays (IGRAs). It has been shown to have better sensitivity for diagnosing active TB compared to the conventional tuberculin skin test (Simsek et al. 2010).</div>\r\n<div>\r\n	The management of ocular TB largely depends on the underlying aetiology. The mainstay of treatment and diagnosis for infective ocular TB (either latent or active) is anti-TB medications, with regular follow-up for monitoring its side effects. Prompt institution of immunosuppressive agents is vision- saving in immune- mediated ocular TB (Gupta et al. 2001). However, this particular case is not a clear cut one, hence we opted to err on the safe side and started anti- TB medication, prior to starting the immunosupressive agent.</div>\r\n<div>\r\n	Clinical features of ocular TB which points towards infective nature are vasculitis, macula oedema, vitritis, choroiditis and anterior uveitis (Alvarez et al. 2009). However, in some cases the vasculitis and macular conditions cannot be ascertained. Ocular coherence tomography and fluorescein angiogram study provide invaluable additional information. Areas of capillary fallout and leaking new vessels are more apparent on angiogram study. Furthermore, macular ischaemia which may occur with or without macular oedema is important as it carries grave visual prognosis.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Ocular tuberculosis may affect individuals despite previous vaccination. The importance of differentiation between immune-mediated and infective TB lies in the management. Fluorescein angiography study and ocular coherence tomography are important diagnostic tools in these cases. High index of suspicion and prompt management is important to save vision, and possibly lives.</div>\r\n', 1),
(1014, 1008, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Ascariasis is a common helminthic infestation in Malaysia, particularly in rural settings. Ascariasis lumbricoides normally lives in the upper small bowel without causing symptoms. Complications arise when these worms migrate into the bile duct (biliary ascariasis) causing biliary colic, cholangitis, acalculous cholecystitis and hepatic abscess. A case of biliary ascariasis presenting as biliary colic is described. Diagnosis was established using both ultrasound of the hepato-biliary system (HBS) and endoscopic retrograde cholangio-pancreatography (ERCP). Endoscopic removal of the worm led to rapid resolution of symptoms.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 49-year-old female of indigenous origin (Orang Asli) first arrived to our district hospital for severe right upper quandrant pain of about two days duration. The pain was paroxysmal, radiating to the back and was accompanied by nausea and vomiting. There was no history of previous attacks of pain, jaundice or fever. On clinical examination, she was febrile (38.5&deg;C) but not jaundiced. The right hypochondrium was tender with mild rigidity. Murphy&rsquo;s sign was positive. Her blood investigation showed her hemoglobinwas 10.5 g/dl and white cell count was 13.5 with predominantly 90% polymorphs, 4% eosinophils and 6% lymphocytes. Serum bilirubin was 36 umol/L. Her liver enzymes were within normal limits. With the provisional diagnosis of acute cholecystitis the patient was managed conservatively with intravenous fluids, iv cefuroxime 1500 mg x 8 hourly and given intravenous analgesia (tramadol 50 mg 6 hourly). The pain subsided for a few hours but recurred with attacks of increasing severity the next day and the patient became mildly jaundiced. Liver function test were remarkable, with raised alkaline phosphatase 300 U/L. In view of these new developments, patient was referred to the nearest tertiary hospital for further investigation and management. Ultrasound of the hepatobiliary system (USG HBS) and CT-scan of the abdomen were proceeded. Her USG revealed a dilated proximal common bile duct and right intra-hepatic ducts with tubular hyper-echoic structure and mild splenomegaly (Figure 1). Her CT-abdomen revealeddistal CBD obstruction with linear density, an image compatible with worm (Figure 2). In view of the above findings, ERCP was performed and resulted in the identification and removal of the worm (Figures 3 and 4). Following the procedure, patient&rsquo;s symptoms subsided and she was subsequently discharged home well with a course of oral antibiotics (Metronidazole 400mg bd) and a stat dose of albendazole (400mg). She remained well on follow-up two weeks later at the district hospital.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Biliary ascariasis can produce a variety of manifestations. It can present with mild or severe recurrent right upper quadrant pain when complicated by secondary infection such as ascending cholangitis (Ibrarullah et al. 2011). Recurrent attacks of cholangitis canmimic cholelithasis. Ascariasis is implicated in cholangio-hepatitis, characterized by cholelithiasis (often without chole-cystolithiasis), biliary duct strictures and hepatic abscess (Misra &amp; Dwivedi 2000). The common duct stones are usually multiple, soft and black. The worm in the CBD may be alive or dead. The degenerated worm or live worm may form a nidus for biliary sand and stones. Super-added bacterial infection also leads to precipitation of bilirubin by enzymatic degradation (Misra &amp; Dwivedi 2000). The worm may migrate into the liver where it excites granuloma formation. Degeneration of the worm in the liver with secondary infection leads to hepatic abscess. In some tropical countries, ascaris liver abscess is commoner than amoebic abscess in children. Ascariasis of biliary tree can cause hemobilia and rarely acute pancreatitis due to obstruction of the ampulla of Vater (Adaletl et al. 2005).</div>\r\n<div>\r\n	Usually one or two worms are present in the biliary channels but sometimes massive invasion can occur. Complications like septicaemia and hepatic abscess are high in massive invasions and require early diagnosis and treatment. The commonest presentation of biliary ascariasis is recurrent biliary colic (95%) which is clinically difficult to distinguish from cholelithiasis (Ortega et al. 2010). Ultrasound is a useful sensitive and specific tool for diagnosing biliary ascariasis with an accuracy of about 86% provided the radiologist is aware and alert to the sonographic appearances. The characteristic sonographic features include: (a) a single or multiple long, linear or curved echogenic structure without acoustic shadowing; (b) a thick, long, linear or curved, non-shadowing echogenic strip containing a central, longitudinal anechoic tube, probably representing the digestive tract of the worm (Ibrarullah et al. 2011). ERCP is an excellent diagnostic and therapeutic tool for biliary ascariasis. At ERCP the roundworms in the biliary tree appear as smooth, long, linear or curved filling defects with tapering ends. Surgery had been the traditional method of removal of worm from the bile duct until the advent of ERCP. Endoscopic removal of worm from the bile duct is safe, minimally invasive and effective (Oswal &amp; Agarwal 2007).</div>\r\n<div>\r\n	Most cases will respond to conservative measures, with the worms returning spontaneously to the intestine. This is enhanced if mebendazole is given orally to deworm the intestine. Mebendazole is preferred as it is poorly absorbed from the intestine. The intestinal worms are killed but the biliary worms are spared and return to the intestine (Ortega et al. 2010). Surgery is reserved for patients who fail to respond to an adequate trial of conservative therapy or show complications like hepatic granuloma and abscess. Other indications for surgery are persistent right upper quandrant pain, severe or increasing jaundice and signs of right upper quandrant peritonitis. Cases treated conservatively should be followed with intravenous cholangiogram. If persistent filling defects are demonstrable, surgery or endoscopic removal is indicated (Sharma et al. 2005).</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1020, 1014, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Emergency contraception (EC) is defined as any drug or device used as an emergency measure to prevent an unplanned pregnancy after unprotected sexual intercourse or a known contraceptive failure (Cheng et al. 2004; Gemzell-Danielsson 2010). Currently, Levonorgestrel-only EC is recognized as a well-established EC (Gemzell-Danielsson 2010). 1.5 mg Levonorgestrel (LNG) should be administered within 72 hours after an unprotected sexual intercourse, either in a single dose or in two doses of 0.75 mg 12 hours apart (Cheng et al. 2004). The effectiveness of LNG as EC had been estimated to be between 57-95% depending on the delay of administration, highest when administered within 24 hours (Mikolajczyk &amp; Stanford 2007; Gemzell-Danielsson 2010; Kozinszky et al. 2011). However, the risk of ectopic pregnancy should be considered following LNG-only EC failure. Numerous cases of ectopic pregnancy after the use of LNG-only EC had been previously reported (Sheffer-Mimouni et al. 2003; Ghosh et al. 2009; Kozinszky et al. 2011). There was a rate of 4.1% ectopic pregnancies following failure of LNG-only EC, which was higher than the incidence of 1.1% to 1.6% in spontaneous pregnancies (Varma &amp; Gupta 2009; Gainer et al. 2001).</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A healthy 18-year-old sexually active nulliparous woman presented to the emergency department with sudden onset of generalised abdominal pain associated with symptoms of severe anaemia at eight weeks period of amenorrhoea. Her menses were regular at 28 to 30 days per cycle. Urine pregnancy test was positive. She had no known risk factor for ectopic pregnancy. She did not practice any method of contraception. She provided a history of unprotected sexual intercourse approximately two weeks after her last normal menstrual period. Following the unprotected sexual intercourse, she took a course of LNG-only EC, 2 doses of 0.75 mg 12 hours apart, with the first dose being administered within 48 hours. The LNG-only EC was purchased over the counter at a local pharmacy. She did not have any sexual intercourse consequently.</div>\r\n<div>\r\n	On assessment, she was in Stage III of hypovolaemic shock with severe pallor. Her blood pressure was 84/39 mmHg and pulse 97/min. Abdomen was distended and guarded. Bimanual examination revealed a uterus of 6 weeks size with bogginess at the pouch of Douglas. Transvaginal ultrasonography revealed an empty uterus with presence of large amounts of haemoperitoneum. She underwent a laparotomy and was found to have a ruptured right tubal pregnancy at the isthmus and a massive haemoperitoneum of 3000 ml. The left tube was normal. A right salpingectomy was performed. She was transfused with three pints of packed cells. The postoperative period was uneventful and she was discharged two days later with a haemoglobin level of 10.5 g/dL. Histology of the surgical specimen confirmed a tubal pregnancy.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The risk factors of tubal ectopic pregnancies include previous ectopic pregnancy, pelvic inflammatory disease, history of pelvic or tubal surgery, intrauterine contraceptive device use, salpingitis isthmica nodosa, infertility, smoking and assisted conception, although up to half of the women with ectopic pregnancies did not have any identifiable risk (Varma &amp; Gupta 2009; Marion &amp; Meeks 2012). However, the failure of emergency LNG-only EC had not been established as a risk factor for tubal ectopic pregnancy despite reported increased rate of ectopic pregnancy following its failure.</div>\r\n<div>\r\n	The mechanism of action of LNG-only EC is not fully known but studies had shown that the mechanism of action and efficacy vary with phase of the menstrual cycle when the LNG EC was administered (Croxatto et al. 2003; Ghosh et al. 2009). The administration of LNG prior to the LH surge was able to inhibit or delay the LH surge, delay follicular development and arrest the unruptured follicles (Croxatto et al. 2003; Gemzell-Danielsson 2010). Administration of LNG also transiently increased the viscosity of cervical mucus, which inhibited the passage of sperms (Noe et al. 2011). However, if taken when LH surge had already started, LNG is not able to prevent ovulation and hence, may be the cause for the reported failures using LNG EC (Croxatto et al. 2004). In addition, the administration of LNG during the peri- and postovulatory periods does not confer any significant change in the endometrial histology or any studied markers of receptivity during the mid-luteal phase at the expected time of endometrial receptivity and implantation (Ghosh et al. 2009; Gemzell-Danielsson 2010). It was concluded that LNG-only EC was highly effective in preventing pregnancy if administered prior to ovulation but if administered after ovulation, it was ineffective in preventing pregnancy, as it did not have any effect on the subsequent reproductive processes, including the implantation of the embryo (Noe et al. 2011).</div>\r\n<div>\r\n	Numerous studies had shown that the single dose of 1.5mg of LNG was equally as efficacious as two doses of 0.75mg LNG as a mode of emergency contraception. In a review of emergency contraception, there was no evidence to indicate any difference in the rates of ectopic pregnancy between these two regimens (Cheng, et al. 2004).</div>\r\n<div>\r\n	Smooth muscle contraction and ciliary activity of the fallopian tube played an important role in the embryo-tubal transport, should fertilisation occur (Shaw et al. 2010). High levels of progesterone in the early luteal phase had been shown to reduce or slow ciliary activity in the fallopian tube and affect the embryo-tubal transport (Paltieli et al. 2000; Gemzell-Danielsson 2010). Following the administration of two doses of 0.75 mg LNG at 12 hours apart or a single dose of 1.5 mg of LNG, the serum level of LNG was high, reaching maximum levels of 25.3 nmol/L and 39.3 nmol/L respectively (Johansson et al. 2002). Theoretically, high levels of LNG would affect the motility of the tubes (Sheffer-Mimouni et al. 2003). LNG was capable of markedly inhibiting the muscular contraction of the fallopian tube based on an in-vitro study (Wanggren et al. 2008). The effect of LNG on the ciliary function and tubal muscular contraction could be the plausible explanation for the occurrence of ectopic pregnancy following emergency contraceptive failure.</div>\r\n<div>\r\n	There were three case reports on the failure of LNG-only EC administered during mid-cycle resulting in tubal ectopic pregnancies (Sheffer-Mimouni et al. 2003; Ghosh et al. 2009; Kozinszky et al. 2011). Camp et al. (2003) reported a rate of 7.4% (n = 21) of ectopic pregnancies from 285 pregnancies after the failure of LNG EC, while Gainer et al. (2001) reported a rate of 4.1% (n = 3) from a total of 73 pregnancies resulting from product failures (Camp et al. 2003; Gainer et al. 2001). However, there was no valid or unbiased assessment done and the authors did not rule out the possibility of unreported normal pregnancies following the product failure (Camp et al. 2003). A systematic review had shown that the rate of ectopic pregnancy following failure of LNG-only EC did not exceed the rate observed in spontaneous pregnancies as EC was effective in reducing risk of unwanted pregnancy and in turn, preventing ectopic pregnancy (Cleland et al. 2010). Nevertheless, the occurrence of ectopic pregnancy following the failure of LNG EC should not be disregarded. In comparison with the intrauterine devide (IUD), a meta-analysis of case-control studies reported that there is no increased risk of ectopic pregnancy with IUD use but a pregnancy with IUD in-situ is more likely to be an ectopic pregnancy (Xiong et al. 1995).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	This case report aims to highlight the occurrence of ectopic pregnancy following the failure of LNG-only EC despite being administered correctly. This is the first case being reported in Malaysia. Numerous case reports and literature had reported ectopic pregnancies after the use of LNG-only EC but it had not been statistically proven to be an established risk factor. This may be due to absence of an accurate method or research to report the exact incidence. However, as recent systematic reviews have failed to prove an increased risk of ectopic pregnancy following LNG-only EC compared to spontaneous conception, the public must not be misled into believing that there is an increased risk of ectopic pregnancy until there is sufficient literature evidence. This is to avoid unnecessary fear to the usage of LNG-only EC causing a rise in occurence of unwanted pregnancies and baby abandonment which had been a disconcerting phenomenon in Malaysia recently.</div>\r\n<div>\r\n	Regulatory bodies in the UK, New Zealand and other countries have made recommendations to &ldquo;consider the possibility&rdquo; of ectopic pregnancy following the failure of LNG-only EC (Cleland et al. 2010). Therefore, both physicians and the public should be made aware of this adverse effect of LNG-only EC use. Prescription leaflets should contain information regarding ectopic pregnancy being one of the side effects of LNG EC use. In addition, physicians and pharmacists who prescribe the EC should educate the users to seek early consultation should there be presence of amenorrhoea for urine pregnancy test, menstrual abnormalities or abdominal pain following the administration.</div>\r\n', 1),
(1026, 1020, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	AML constitutes 15-20% of all acute leukaemia cases in children 15 years and below. According to the National Cancer Registry of Cancer Incidence in Malaysia 2007, the incidence of AML is 2.8 per 100,000 population. The age specific cancer incidence per 100,000 population for 0-9 years of age in Malaysia is 3-6 for males and 2-4 for females.</div>\r\n<div>\r\n	Acute megakaryoblastic leukemia (AML M7) is an uncommon subtype of AML, occuring in approximately 10% of paediatric and 5% of adult AML (Bernstein et al. 2000, Carroll et al. 1991). It was reported that its incidence is highest among infants with Down syndrome and is associated with mediastinal germ cell tumors (Swerdlow et al. 2008).</div>\r\n<div>\r\n	The t(1;22) is an uncommon abnormality in AML, constituting less than 1% of all AML cases. It is found most commonly among non-Down syndrome infants, predominantly in females. AML with t(1;22) is a de novo AML, restricted to infants and young children, with most cases occuring in the first six months of life. It is associated with t(1;22)/RBM15-MKL1 resulting in expression of the one twenty-two megakaryocytic acute leukaemia (OTT-MAL) fusion gene (Carroll et al. 1991; Ma et al. 2001; Mercher et al. 2009). The expression of OTT-MAL fusion gene results in abnormal megakaryocyte development. The megakaryopoiesis changes include enhancement in the cells self-renewal ability. Apart from that there is activation of recombination signal binding protein for imunoglobulin J kappa region (RBPJ)-mediated transcription (Mercher et al. 2009). It is suggested that prenatal genetic factors are involved in leukaemogenesis in these AML M7 infants having t(1;22) (Carroll et al. 1991). These infants usually present with huge hepatosplenomegaly, anaemia, thrombocytopenia and moderate elevation of the white blood cell count. At presentation, they commonly come with cytopenias, particularly thrombocytopenia, however some may have normal or increased in platelet count.</div>\r\n<div>\r\n	The megakaryoblasts show similar morphological features in all AML-M7 with or without t(1;22). The blasts are heterogenous with some undifferentiated blast cells resembling lymphoblasts. These megakaryoblasts are medium to large in size with slight irregular or indented nucleus with fine reticular chromatin and prominent nucleoli. They have basophilic cytoplasm, often agranular and some showed distinct blebs or pseudopod formation (Swerdlow et al. 2008). The bone marrow biopsy may show hypercellular marrow with evidence of fibrosis (Carroll et al. 1991). These marrow fibrosis may be attributable to the secretion of fibrogenic cytokines by the megakaryoblasts (Carroll et al. 1991).</div>\r\n<div>\r\n	Immunophenotypically, the megakaryoblasts express one or more platelet glycoprotein CD41 (glycoprotein IIb/IIIa), and/or CD61 (glycoprotein IIIa) and other myeloid markers CD13 and CD33. The more mature platelet-associated marker CD42 (glycoprotein Ib) is less frequently expressed. CD34, CD45, MPO and HLA-DR are usually negative (Swerdlow et al. 2008).</div>\r\n<div>\r\n	Therapy of AML generally consists of aggressive multi-drug chemotherapy regimens consisting of cytarabine, anthracyclines (daunorubicin, idarubicin, and mitoxantrone) and etoposide (Bain 2007) aimed to eliminate the leukaemic cells and achieve a complete haematological remission (CR). Our centre used the AML 15 protocol similar to a study trial conducted in Birmingham.</div>\r\n<div>\r\n	AML M7 has a better prognosis among Down&rsquo;s syndrome infants, with a four year event free survival of 73%. The prognosis in both children and adults with AML M7 with t(1;22) is poor, primarily attributable to a resistant disease (Bain, 2007). Approximately 50% of patients will obtain an initial remission with cytarabine and anthracycline but with only 10.5 months median survival (Carroll et al. 1991). Bone marrow transplantation is indicated in all cases of AML M7 with t(1;22) (Lion et al. 1992). However, long term survivors following intensive therapies have been reported (Reinhardt et al. 2005).</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 9-month-old baby girl presented to our centre with pallor, lethargy,abdominal distension, loose stool, vomiting and reduced oral intake of two weeks duration. On physical examination, she was noted to be underweight with failure to thrive, pale and tachypnoeic. There were huge hepatosplenomegaly and multiple lymphadenopathies involving the cervical, axillary, inguinal, and occipital region. Areas of ecchymoses were noted on her back. A soft systolic murmur was present with displaced apex beat at the 6th intercostal space, 1 cm lateral to the mid-clavicular line.</div>\r\n<div>\r\n	Full blood count showed severe anaemia with haemoglobin of 2.6 g/dL, leukocyte count of 19.9 x 109/L, and platelet count of 23 x 109/L. The full blood picture revealed severe anaemia, ovalostomatocytosis, thrombocytopenia with leukoerythroblastic picture and presence of blasts (Figure 1, 2). The bone marrow aspirate showed presence of 60% blasts which are heterogenous in size. The blasts have high nucleo-cytoplasmic ratio, immature nuclei, inconspicuous nucleoli, and abundant basophilic cytoplasm. Some of the blasts have cytoplasmic blebs. No Auer rods or granules are noted within the cytoplasm (Figure 3). The peroxidase stain was negative (Figure 4).</div>\r\n<div>\r\n	Immunophenotyping of the bone marrow aspirate performeddemonstrated an abnormal population of blasts expressing CD117, CD13, CD33(dim) and CD61 and negative for other B and T cell markers consistent with acute megakaryoblastic leukaemia. The trephine biopsy was inconclusive as the sample was unsatisfactory. Interestingly, the cytogenetic study showed translocation between chromosome 1 and chromosome 22 (t(1;22)(p13;q13)) in sixteen metaphase spreads (Figure 5). Flourescent in situ hybridisation (FISH) analysis showed no visible BCR/ABL, TEL/AML1, or MLL gene rearrangement. The serum lactate dehydrogenase (LDH) was markedly elevated (7916 U/L).</div>\r\n<div>\r\n	She received multiple transfusion of packed cells, platelets and was started on the ADE chemotherapy protocol which consist of cytosine arabinoside (Ara-C), daunorubicin and etoposide. She developed haematuria following platelet transfusion which after ultrasound of the kidney revealed mild to moderate hydronephrosis with hydroureter.</div>\r\n<div>\r\n	During the course of chemotherapy, she developed neutropenic sepsis with bronchopneumonia complicated with acute respiratory distress and needed intensive care monitoring, continuous positive airway pressure (CPAP) and inotrope support. She was ventilated for 12 days and was given broad spectrum antibiotics Meropenem and Amphocil, as well as granulocyte colony-stimulating factor (G-CSF) to increase the neutrophil counts. Her condition improved and was allowed for discharge after one week recovery. The parents were informed to bring her for follow up within a week for further management. Unfortunately, her parents defaulted the follow-up. A few months later the child was brought in with relapsed acute myeloid leukaemia. She was re-started on the ADE protocol and completed the cycles. Currently she is well and is on maintenance chemotherapy with oral thioguanine and sub-cutaneous Ara-C.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Acute megakaryoblastic leukaemia (AML M7) is an uncommon subtype of AML, comprising approximately 10% of paediatric and 5% of adult AML (Bernstein et al. 2000; Carroll et al. 1991). It has a bimodal age distribution that peaks in early childhood, normally in children less than 3 years of age and adulthood (Gewirtz et al. 1989; Ribeiro et al. 1993). In a large series of childhood AML from a single institution, 14.6% of all cases of AML were identified as AML M7 (Athale et al. 2001). The incidence of AML M7 is much higher than what was previously reported. This could be attributed to the complexity of its clinical presentation and difficulty in obtaining and interpretating the bone marrow samples for diagnostic testing (Ravindranath et al. 1992). Thus, many of these AML M7 cases may have been diagnosed as undifferentiated leukaemia, myelodysplastic syndrome, myelofibrosis or other disease (Athale et al. 2001). Currently, the improvement in knowledge on the morphology, cytochemistry, and immunophenotyping of the megakaryoblasts, together with the advancement in cytogenetic testing have allowed a more accurate and reliable diagnosis of AML M7 as well as determination of the character and prognosis of this disease.</div>\r\n<div>\r\n	In children, the incidence of AML M7 is highest among infants with Down syndrome with trisomy 21 and also in children with mediastinal germ cell tumors (Swerdlow et al. 2008).</div>\r\n<div>\r\n	In contrast, AML M7 in infants without Down syndrome is found to be exclusively associated with t(1;22)/RBM15-MKL1 resulting in fusion of the OTT(RBM15) gene on 1p13 to the MAL(MLK1) gene on chromosome 22, leading to the OTT-MAL fusion gene on the derivative 22 (Swerdlow et al. 2008; Carroll et al. 1991; Ma et al. 2001; Mercher et al. 2009). It is an uncommon subtype in AML, constituting less than 1% of all AML cases and found most commonly among non-Down syndrome infants, predominantly in females. Other cytogenetic abnormalities that may be present in these non-Down syndrome infants with acute megakaryoblastic leukaemia includes inv(3)(q21;q26), which is not specific to AML-M7 and may be seen in other types of AML (Edmond et al. 2001). The insertion(12p) can also be found among patients with AML-M7 but it is more commonly associated with mediastinal germ cell tumours (Edmond et al. 2001).</div>\r\n<div>\r\n	AML with t(1;22) is a de novo AML, restricted to infants and young children, with most cases occuring in the first six months of life. It is suggested that prenatal genetic factors are involved in leukaemogenesis in these AML M7 infants with t(1;22) (Carroll et al. 1991). These infants usually present with huge hepatosplenomegaly, anaemia, thrombocytopenia and moderate leucocytosis. Cytopenias, particularly thrombocytopenia, are common at presentation but the platelet count can be normal or even increased in some cases. Characteristically, there is no preceeding myelodysplasia, or history of transient leukaemoid reaction in these infants (Verschuur et al. 2004). These features were well illustrated as reported in our case.</div>\r\n<div>\r\n	The morphological features of the blasts presented in our patient were similar to that described for AML M7 with or without t(1;22). In addition, the stromal pattern of bone marrow infiltration by these megakaryoblasts may mimic a metastatic tumour. The blasts are usually myeloperoxidase negative, but are positive for acid phosphatase and alpha-naphthyl acetate esterase (sodium fluoride resistant).</div>\r\n<div>\r\n	Immunophenotypically, the megakaryoblasts express platelet glycoproteins, namely CD41 (glycoprotein IIb/IIIa), and/or CD61 (glycoprotein IIIa), together with the myeloid markers of CD13 and CD33. CD34, CD45, MPO and HLA-DR are usually negative (Swerdlow et. al 2008). The immunophenotyping from our patient showed expression of CD117, CD13, CD61 and CD33(dim) which are consistent with the diagnosis of AML M7.</div>\r\n<div>\r\n	Based on the general principle of leukaemia therapy, there are multiple chemotherapy protocols available from different centres conducting these trial studies. Among them is the AML 15 trial study conducted in Birmingham in which our patient&rsquo;s treatments were based upon. The ADE 10+3+5 protocol from the AML 15 trial were used. Each course of chemotherapy results in a temporary bone marrow suppression, leading to prolonged anaemia, leukopenia, neutopenia and thrombocytopenia. The prolonged myelosuppression often render the patients susceptible to opportunistic bacterial, fungal or viral infection, which can be life-threatening. Moreover, the chemotherapy courses can also cause varying degree of mucositis, which is caused by the cytotoxic effect of the chemotherapy agents on the epithelium of the intestinal tract, thus requiring various supportive measures.</div>\r\n<div>\r\n	The prognosis in both children and adults with this form of leukemia is poor, primarily attributing to its resistant disease (Bain&lsquo; 2007). Approximately 50% of patients will obtain an initial remission with cytarabine and anthracycline but the median survival is only 10.5 months (Carroll et al. 1991). AML M7 has a better prognosis among Down&rsquo;s syndrome infants, with a 4 year event free survival of 73% and bone marrow transplantation is indicated in cases of AML M7 with t(1;22) (Lion et al. 1992). However, long term survivors following intensive therapies have been reported (Reinhardt et al. 2005).</div>\r\n<div>\r\n	In conclusion, AML M7 is a heterogenous disease affecting the two extreme ages and carries a poor prognosis, contributed by the resistant nature of the disease. AML M7 commonly affect infants with Down&rsquo;s syndrome or associated with t(1;22) in those with non-Down syndrome. Early recognition and diagnosis of AML M7 especially to identify the subtype with t(1;22) is very crucial to stratify these patients to the more intensive management in order to achieve good response to treatment and improving their overall survival. We highlighted a case of an infant with a rare subtype of AML M7 with t(1;22) who had successfully undergone an intensive series of chemotherapy and currently well on maintenance treatment.</div>\r\n', 1),
(1027, 961, NULL, NULL);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1029, 1022, '<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Obesity had been considered as one of the risk factors for many medical illnesses such as ischaemic heart disease, hypertension, and diabetes mellitus (WHO). Apart from that, obesity could also affect the psychosocial well-being of obese people. &nbsp;Many obese people have lower self-esteem which might predispose them to the development of depression (Atlantis and Baker, 2008). The stigma that the society holds on the obese people could be detrimental to their psychosocial well-being which may lead to the development of depression. &nbsp;Hence, this research aims to find out the association between obesity and depression. &nbsp;</div>\r\n<div>\r\n	&nbsp;A study by Pesa, Syre, and Jones (2000) in Norway on 3197 female adolescents to assess the importance of body image among the female adolescents &nbsp;showed a relationship between depression and body weight. In many cases, this relationship is influenced by factors such as self-esteem and body image. It strongly suggested that obesity was a risk factor for social problems and implied that an oversized female adolescent usually felt less confident in her social world among family and friends.</div>\r\n<div>\r\n	Obese people were always stigmatized and discriminated by people surrounding them especially in the working environment, social setting, school and during interactions with healthcare professionals (Foster et al, 2003). Some people enjoy criticizing obese people which may leave a negative impact on these people. &nbsp;The way they &nbsp;respond to such remarks would depend very much on their upbringing and character. &nbsp;Some may be determined to lose their weight and start dieting and exercising, while some may respond negatively and resort to impulsive eating. &nbsp;</div>\r\n<div>\r\n	The main objective of this present study was to determine the prevalence of depression among obese people working in Malaysia. It explored the psychological aspect of obese people and the perception the others had on them. &nbsp;</div>\r\n<div>\r\n	Obesity is defined by Body Mass Index (BMI). According to World Health Organization classification of obesity, &ge; 30kg/m2 was considered as obese. Obesity is further divided into class I, class II and class III. For class I obesity, it ranged from 30.0 to 34.9; for class II, it ranged from 35.0 to 39.9; and for class III, it was more than 40.0 (WHO, 1998)</div>\r\n<div>\r\n	&nbsp;In a study conducted by Lehrke and Laessle (2002), obesity was defined as an increase in fatty tissue that exceeded a certain percentage of the total body weight. This had shown to have a strong association with certain health risks and even increase in mortality rate. Based on this study, men with 25% body fat and women with more than 30% body fat were considered obese.</div>\r\n<div>\r\n	&nbsp;Obesity among &nbsp;the &nbsp;Malaysians is on the rise each year. According to the National Health and Morbidity Survey, NHMS II (1996), the prevalence of overweight is 20.7% while obese adults are 5.8% in Malaysia. These percentages were higher compared to other countries in Asia such as Japan, Thailand, and China (Khor, 1997). NHMS III (2006) showed that 29.1% of the adult population studied were found to be overweight (BMI 25.0 to 29.9).&nbsp;</div>\r\n<div>\r\n	&nbsp; The prevalence of obesity (BMI &gt;30) was reported to be 14.0%. The proportion of the adult population who were overweight and obese was 43.1%. It can be assumed that almost half of the adult population in the country could be overweight or obese. This rate of occurrence of overweight and obesity is much higher than the rates found in the Second National Health and Morbidity Survey (NHMS II) which was carried out in 1996. This could be due to the shift towards westernized dietary pattern and physical inactivity which led to the increased rate of obesity and other nutrition-related non-communicable diseases among the population, including Malaysians (Tee, 1999).</div>\r\n<div>\r\n	&nbsp; Obesity has become one of the most important public health issues (World Health Organization-Food and Agriculture Organization, 2002) and is known to be one of the hazards which may &nbsp;lead to &nbsp;health problems later in life. Obesity was highly associated with &nbsp;morbidities such as Type II diabetes, hypertension and ischemic heart diseases. This will be the greatest threat to human lifestyle and health in a developing country and may cause a big burden worldwide especially to the health care centres, Howie, Sloboda, Kamal, Vickers (2009). In many countries, rapid growth of the economic status had brought changes in the population&rsquo;s lifestyle and dietary intakes. These changes has also contributed to increasing prevalence of obesity and other diet-related chronic diseases (WHO, 1998).&nbsp;</div>\r\n<div>\r\n	&nbsp;Rampal, Rampal, Khor, Zain, Ooyub, Rahmat, Ghani and Krishnan (2007) conducted a national study on the prevalence of obesity among 16,127 Malaysian; found that subjects with a body mass index &ge; 30 kg/m2 were labelled as obese. The results showed that the overall national prevalence of obesity among Malaysians aged 15 years old and above was 11.7% (95% CI = 11.1 - 12.4%). &nbsp;The prevalence of obesity was significantly higher in females which was 13.8% compared to 9.6% in males (p&lt; 0.0001). &nbsp;Prevalence of obesity was highest amongst the Malays and Indians, 13.6% and 13.5% respectively followed by the indigenous group of &ldquo;Sarawak Bumiputra&rdquo; and the Chinese, 10.8% and 8.5% respectively. The indigenous group of &ldquo;Sabah Bumiputra&rdquo; had the lowest prevalence which was 7.3%. &nbsp;In conclusion, the prevalence of obesity amongst those aged &ge; 18 years old had markedly increased by 280% since the last National Health and Morbidity Survey in 1996.</div>\r\n<div>\r\n	Obese people were always being stigmatized and discriminated by people surrounding them especially in the workplace, social setting, school and interactions with health professionals. Most people used to tease and embarrass the people who were obese due to their physical appearance. The fact that obese people were being discriminated was further supported by a study carried out by Foster et al., (2003). Almost 50% of physicians viewed obese patient as ugly, unattractive, awkward, lack of self-control and lazy. This thought may lead to an increased distance between physicians and patients and may indirectly influence the treatment compliance</div>\r\n<div>\r\n	Ogden and Clementi (2010) found that participants in their study termed the emotional consequences of their weight as &ldquo;miserable&rdquo;, &ldquo;depressing&rdquo;, &ldquo;disgusting&rdquo;, &ldquo;killing myself &rdquo;, &ldquo;ashamed&rdquo;, &ldquo;guilty&rdquo;, and &ldquo;hatred&rdquo;. Most of the remarks were negative and involved a negative sense of self, negative affect, a sense of dissociation and concerns about their health. However, only a small minority felt that their weight had no or little impact on their life. &nbsp;(Carpenter,Hasin, Allison, Faith, 2000; Strauss &amp; Pollack, 2003 stated that obesity &nbsp;has significant relation with depression, suicidal thoughts, and even suicidal attempts</div>\r\n<div>\r\n	&nbsp;Pesa, Syre, and Jones (2000), had conducted a study to assess the importance of body image especially among the female adolescents. The result showed a relationship between depression and body weight. In many cases, this relationship was influenced by factors such as self-esteem and body image. It strongly suggested that obesity is a risk factor for social difficulty and implied that an overweight female adolescent might feel less confident in her social world&mdash;family, community, or school.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	&nbsp;A quantitative, cross-sectional study was conducted using a convenient sampling method among patients and staff who were considered obese according to the BMI results.The study was carried out on all patients attending clinic sessions in various clinic in Universiti Kebangsaan Medical Centre. The &nbsp;criteria of patients for this study was: any obese patient attending as outpatients, aged between 25 to 45 and with a BMI of 30 and above. This study was carried out only on people who were holding a job and should be Malaysians</div>\r\n<div>\r\n	&nbsp; &nbsp;Data was collected from 60 patients who were randomly selected and only those who were willing to participate were included. The respondents had to complete a consent form and the respondent&rsquo;s weight and height were taken to calculate their BMI prior to data collection.</div>\r\n<div>\r\n	A structured questionnaire that consisted of five sections was used to collect data. The first part of the questionnaire was on demographic data. Then the respondent&rsquo;s height and weight were taken for BMI calculation. The second part of the questionnaire consisted of ten frequency questions and three open ended questions that allowed the respondent to assess the insight and the respondents feeling on been obese.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A pilot study was carried out on five respondents who were not included in the study after modification to the third, forth and fifth part of the questionnaire and the cronbach alpha reliability results were as shown below.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;The third part of the questionnaire was modified from Eating Style Questionnaire (ESQ) which had earlier being used by Hargreaves, schlundt, Buchowski, Hardy, Rossi, Rossi (1999). This questionnaire was modified a little and the Cronbach alpha showed a reliability of 0.869 suggesting a good internal consistency.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; For the fourth part, a &nbsp;Body Image Avoidance Questionnaire (BIAQ) was used and the Cronbach alpha suggested an internal consistency of 0.65. Dr. Goldberg&rsquo;s Depression Test was used to test whether the patient was suffering from any depression. The translated questionnaire had Cronbach alpha reliability of 0.818</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;A total of 70 people completed the questionnaire. &nbsp;However, only 60 respondents fulfilled the criteria. All the data from 60 respondents was analysed using SPSS version 20. For statistical analysis, chi-square test and one-way ANOVA were used.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Ethical consideration</div>\r\n<div>\r\n	&nbsp;The study was carried out after approval was granted by the Universiti Kebangsaan Malaysia Medical Centre Research and Ethics committee. The clinic heads were also informed of the study.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	&nbsp;There were 60 subjects who participated in this study. There were 56.7% (n=34) males and 43.3% (n=26) females. Their age ranged from 25 to 45 years old. 49 (81.7%) of subjects are Malays, 6 (10%) are Indians and 5 (8.3%) are Chinese. 73.3% (n=44) of subjects are married while 26.6 % (n=16) are single or widowed. All of them were working Malaysian citizens. Their BMI were all above 30 with the lowest value was 30.04.</div>\r\n<div>\r\n	&nbsp;Forty-five &nbsp;(n=27) had positive family history of obesity while 55% (n=33) do not. 58.3% (n=35) respondents had moderate daily lifestyle, 26.7% (n=16) had a sedentary lifestyle while a small number of 15% (n=9) respondents had an active lifestyle. 45% (n=27) respondents started to be obese during their adulthood, 21.3% (n=13) started during late teens, 16.7 %(n=10) early teens, 13.3% (n=8) started in their thirties while 3.3% (n=2) started in their forties.</div>\r\n<div>\r\n	&nbsp;Respondents who scored total amount of 10 and above in Goldberg depression test had higher possibilities of depression. &nbsp;This total was taken because this tool is solely for depression screening. Among the 60 obese respondents, a total of 14 (23.33%) scored more than 10. Eleven of them were male and three were female. However, association between gender and depression was found statistically not significant (X2 =3.568, p = 0.059). The association between class of obesity with depression was also found &nbsp;to be statically insignificant with a p value of more than 0.05 (p= 0.772).</div>\r\n<div>\r\n	&nbsp;Respondents who scored a total mark of 18 and above in Eating Style Questionnaire were considered taking active measures to control their body weight because they tried &nbsp;practice healthy eating habits. Out of the 30 respondents (50%) who took precautions in their daily food intake, 19 of them were not depressed. Chi-square test indicated that there was association between depression and the active measures taken to control weight (X2=5.963, p=0.015).&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	&nbsp; Dragan and Akhtar-Danesh had conducted a study in the year of 2007 on 36,984 respondents in Canada. It stated that the association between depression and BMI is gender dependent. In males, the higher BMI may result in a more severe form of depression while in females the relation may not be the same. &nbsp;Pesa, Syre, and Jones (2000), who conducted a study to assess the importance of body image among female adolescent found a significant relationship between depression and body weight among adolescent female. The present study showed no association between gender and depression. (X2= 0.3568,p=0.059) This could be due to the small sample size which was inadequate to show the significance. &nbsp;</div>\r\n<div>\r\n	&nbsp;Atlantis and Baker (2008) have also strongly suggested that obesity may increase the risk of depression. However, the present study did not represent this as not all obese people were concerned about their weight. Some of them were quite contented with their body image. The present result showed that the prevalence of depression among the obese population was only 23.33%. &emsp;</div>\r\n<div>\r\n	&nbsp;Higher BMI was associated with more severe form of depression. The more severe form of depression may result in lesser weight gain (Dragan &amp;Akhtar-Danesh, 2007). The &nbsp;study revealed that obese people who were depressed, 23.33% (n=14) were associated with more active measures to reduce their weight compared to those who were not depressed, 76.67% (n=46). However, the exact value of weight loss between the depressed and the non-depressed could not be measured in the present study. The measures to reduce weight could be determined by the results but the amount of weight loss could not be quantified. &nbsp;From this study, we could not determine if the weight loss is secondary to depression or because of their initiative and self-awareness.</div>\r\n<div>\r\n	&nbsp;Obesity also has close relation with depression, suicidal thoughts, and even suicidal attempts (Carpenter,Hasin, Allison,&amp; Faith, 2000; Strauss &amp; Pollack, 2003). Sympathising with obese people is not a proper way to change the negative attitude of obese people (Gapinski, Schwartz, &amp; Brownell, 2003; Teachman et al., 2003). From the present study, it was obvious that only a minority of the obese people were likely to become depressed (23.33%, n=14). Although this study showed that not all obese people were likely to be depressed (76.7%, n=46), the chances for them to get depressive symptoms was still possible (Atlantis and Baker, 2008). Without proper management and prevention, this can lead to unwanted health outcome.</div>\r\n<div>\r\n	&nbsp;Obese people were particular about what they consumed and this showed that they were aware of their problems and took active measures to reduce their weight. Obese people took the effort to reduce fat intake by eating less red meat, snack food, were particular about food dressings and dairy products. They also took lots of fruits, vegetable, cereals, pastas, and low-fat food (Hargreaves et al., 2003). Obese people were aware about the importance of dietary intake in weight management. Reduction of body weight requires reduced calorie intake to achieve slow but progressive weight loss (Lang and Froelicher, 2006). Based on the Eating Style Questionnaire (ESQ), there was some association between being obese and eating style. The study indicated that 50% (n=30) chose to eat something of lower fat content and calories.</div>\r\n<div>\r\n	The other problem with obese people was that, they felt that they were being discriminated by the society and the society did not really tolerate and appreciate the presence of obese people (Andreyeva, 2008). Negative mindset on weight-based stereotypes, low-weight self-efficacy, and negative self-body image could increase the risk of depression among obese people (Atlantis and Baker, 2008). Media advertisement of obese people had also created a misconception among themselves (Greenberg, Eastin, Hofshire, Lachlan, &amp; Brownell, 2003). Obese patients faced problems when they had appointment with the physician, where they received less appointment and practically were allowed limited time b with the physicians or doctors. They claimed that they were often been criticised by the physicians (Hebl &amp; Xu, 2001). This thought may lead to further distancing between physicians and patient and thus will indirectly influence the management and treatment compliance. Based on Body Image Avoidance Questionnaire (BIAQ) that was being used in the study, 56.7% (n=34) showed positive attitude towards themselves. The present &nbsp;study too informed that not all obese people felt discriminated when compared; some of them felt contended with their body shape. Apart from that, they thought that the people surrounding them were quite concerned about their weight as it may lead to lots of medical illness.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CONCLUSION AND IMPLICATIONS</div>\r\n<div>\r\n	&nbsp;The prevalence of depression among obese people may be a small percentage but it should &nbsp;not be overlooked. The shame and self-worthlessness feeling that they kept within themselves may someday erupt and have negative outcome on their physical and emotional health. &nbsp;However, using a set of questionnaire in obtaining the information is not appropriate as many respondents responded with superficial feeling. A qualitative study would have been more appropriate as the respondents would have been able to reveal their actual feeling of been obese.</div>\r\n<div>\r\n	&nbsp;It is hoped that through this research, the public will understand the pain and struggle of the obese people. They need moral support to help them in managing this kind of disability and improving their general health.The present study did not show much distress among the obese people some were in fact quite contented with their size.</div>\r\n<div>\r\n	&nbsp;People with a positive mindset and want to reduce their body weight will be able to deal with this disability better. The number of depressed people due to obesity in the present study maybe &nbsp;small (23.33%) but without proper management and prevention, the depressive symptom can lead to unfavourable health outcome. Early referrals must be made to prevent further catastrophe.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	&nbsp; The approval to conduct the study was provided by the UKMMC Medical Research and Ethics Committee with grant number FF-447-2011.We would like to thank the director of the hospital for allowing us to conduct the study in the hospital. &nbsp;We would also like to extend our gratitude specially to our respondents who willingly participated in the study despite its sensitive nature.</div>\r\n<div>\r\n	<div>\r\n		REFERENCES</div>\r\n	<div>\r\n		Atlantis, E. and Baker M. 2008. Obesity effects on depression: systematic review of epidemiological studies. International J of Obesity. 32: 881&ndash;891; doi:10.1038/ijo.2008.54</div>\r\n	<div>\r\n		Bray, G.A. 2004. Medical Consequences of Obesity. The J of Clinical Endocrinol &amp; Metab. 89(6):2583&ndash;2589&nbsp;</div>\r\n	<div>\r\n		Cali, A.M. and Caprio, S. 2008. Obesity in Children and Adolescents. J Clin Endocrinol Metab. 93: S31&ndash;S36</div>\r\n	<div>\r\n		Cawley, J. and Meyerhoefer, C. 2010. The Medical Care Costs of Obesity: An Instrumental Variables Approach. NBER Working Paper. No. 16467 October 2010. .JEL No. D62,G22,H23,I1</div>\r\n	<div>\r\n		Cheong, S.M., Kandiah, M., Chinna, K., Chan, Y.M., Saad, H. 2010.Prevalence of Obesity and Factors Associated with it in a Worksite Setting in Malaysia. J of Community Health. Volume 35(6): 698-705, DOI: 10.1007/s10900-010-9274-1</div>\r\n	<div>\r\n		Conradt, M. 2008. &nbsp;Associations Among Obesity-Related Guilt, Shame, and Coping. J of Clin Psycho. Volume 64(10):1129&ndash;1144.</div>\r\n	<div>\r\n		deZwaan, M., Petersen, I., Kaerber, M., Burgmer, R., Nolting, B., Legenbauer, T., Benecke, A., Herpertz, S. 2009. Obesity and Quality of Life: A Controlled Study of Normal-Weight and Obese Individuals. Psychosom. 50:5, September-October 2009</div>\r\n	<div>\r\n		Dehghan, M., Akhtar-Danesh, N., Merchant, A.T. 2005.Childhood obesity, prevalence and prevention. Nutrition J. 4(240.</div>\r\n	<div>\r\n		Dragan, A. and Akhtar-Danesh, N. 2007. Relation between body mass index and depression: a structural equation modeling approach. doi:10.1186/1471-2288-7-17</div>\r\n	<div>\r\n		Elias, M.F., Elias, P.K., Sullivan, L.M., Wolf, P.A., D&#39;Agostino, R.B. 2002. Lower cognitive function in the presence of obesity and hypertension: the Framingham heart study. International J of Obesity. 27: 260&ndash;268.</div>\r\n	<div>\r\n		Foster, G.D., Wadden, T.A., Makris, A.P., Davidson, D., Sanderson, R.S., Allison, D.B., &nbsp;Kessler, A. 2003. Primary Care Physicians&#39; Attitudes about Obesity and Its Treatment.Obes Res. 2003 Oct;11(10):1168-77.</div>\r\n	<div>\r\n		Freedman, D.S., Wang, J., Thornton, J.C., Zuguo, M., Pierson Jr, R., Dietz, W.H., Horlick, M. 2008. Racial/ethnic Differences in Body Fatness Among Children and Adolescents. Obesity (Silver Spring). 2008 May;16(5):1105-11.</div>\r\n	<div>\r\n		Freedman, D.S., Zuguo, M., Srinivasan, S.R., Berenson, G.S., Dietz, W.H. 2007. Cardiovascular risk factors and excess adiposity among overweight children and adolescents: the Bogalusa Heart Study. J of Pediatrics.150(1):12&ndash;17.</div>\r\n	<div>\r\n		Hassapidou, M., Fotiadou, E., Maglara, E., Papadopoulou, S.K. 2006. Energy intake, diet composition, energy expenditure and body fatness of adolescents in Northern Greece. Obesity (Silver Spring). May 2006;14(5):855-62.</div>\r\n	<div>\r\n		Kee, C.C., Jamaiyah, H., Noor Safiza, M.N., Geeta, A., Khor, G.L., Suzana, S., Jamalludin, A.R., Rahmah, R., Ahmad, A.Z., Ruzita, A.T., Wong, N.F., Ahmad Faudzi, Y. 2006. Abdominal Obesity in Malaysian Adults: National Health and Morbidity Survey III (NHMS III, 2006). Malaysian J of Nutrition.</div>\r\n	<div>\r\n		Khambalia, A.Z and Seen, L.S. 2010. Trends in overweight and obese adults in Malaysia (1996&ndash;2009): a systematic review. Obese Review. June 2010;11(6):403-12.</div>\r\n	<div>\r\n		Kipping, R.R., Jago, R., Lawlor, D.A. 2008. Obesity in children. Part 1: Epidemiology, measurement, risk factors, and screening. BMJ. 2008, 337:a1824doi:10.1136/bmj.a1824</div>\r\n	<div>\r\n		Kleiser, C., Rosario, A.S., Mensink, G.B.M., Reinhild, P., Kurth, B. 2009. Potential determinants of obesity among children and adolescents in Germany: results from the cross-sectional KiGGS study. BMC Public Health. 9:46 doi:10.1186/1471-2458-9-46</div>\r\n	<div>\r\n		Lang, A. andFroelicher, E. S. 2006. Management of overweight and obesity in adults: behavioral intervention for long-term weight loss and maintenance. European J of Cardiovascular Nursing.doi: 10.1016/j/ejenurse.2005.11.002</div>\r\n	<div>\r\n		Lang, A., and Froeliche, E.S. 2006. Management of overweight and obesity in adults: Behavioral intervention for long-term weight loss and maintenance. Eur J Cardiovasc Nurs. June 2006;5(2):102-14.&nbsp;</div>\r\n	<div>\r\n		Loo, T.H., Maniam, T., Ainsah, O. 2011. Psychiatric Morbidity, Personality Profile and Saliva Cortisol Levels in Overweight and Obese Patients Referred to Dietician Clinics in UKMMC. Malaysian J of Psychiatry. MJP-02-01-11</div>\r\n	<div>\r\n		Mellor, D., Ricciardelli, L.A., McCabe, M.P., Yeow, J., Mamat, N.H., Mohd, N. F. 2010. Psychosocial Correlates of Body Image and Body Change Behaviors Among Malaysian Adolescent Boys and Girls. Sex Roles. 63 (5-6): 386-398, DOI: 10.1007/s11199-010-9812-4</div>\r\n	<div>\r\n		Moderate Obesity Takes Years Off Life Expectancy, Science Daily. (Mar. 20, 2009)</div>\r\n	<div>\r\n		Nishitani, N. and Sakakibara, H. 2006. Relationship of obesity to job stress and eating behavior in male Japanese workers. International J of Obesity. (2006): 30, 528&ndash;533. doi:10.1038/sj.ijo.0803153;</div>\r\n	<div>\r\n		&nbsp;Ogden, J. and Clementi, C. 2010. The Experience of Being Obese and the Many Consequences of Stigma.Hindawi Publishing Corporation. Journal of Obesity Volume 2010: Article ID 429098, 9 pages. doi:10.1155/2010/429098</div>\r\n	<div>\r\n		Orzano, A.J and Scott, J.G. 2004. Diagnosis and Treatment of Obesity in Adults: An Applied Evidence-Based Review. The J of the American Board of Family Practice. 17:359-369</div>\r\n	<div>\r\n		Pesa, J.A., Syre, T.R., Jones, E. 2000. Psychosocial differences associated with body weight among female adolescents: the importance of body image. J of &nbsp;Adolescent Health. 26: 330-337.</div>\r\n	<div>\r\n		Puhl, R.M. and Heuer, C.A. 2009. The Stigma of Obesity: A Review and Update. Obesity. 2011; doi:10.1038/oby.2008.636</div>\r\n	<div>\r\n		Rampal, L., Rampal, S., Khor, G.L., MdZain, A., Ooyub, S., Rahmat, R., Sirajoon, N.G., Krishnan, J. 2007.A national study on the prevalence of obesity among 16,127 Malaysian Asia Pac J Clin Nutr. 2007; 16 (3):561-566.</div>\r\n	<div>\r\n		Simon, G.E., Von Korff, M., Saunders, K., Miglioretti, D.L., Crane, P.K., van Belle, G., Kessler, R.C. 2006. Association Between Obesity and Psychiatric Disorders in the US Adult Population. Arch Gen Psychiatry. 2006; 63:824-830</div>\r\n	<div>\r\n		Wang, S.S., Brownell, K.D., Wadden, T.A. 2004.The influence of the stigma of obesity on overweight individuals. Int J Obes Relat Metab Disord. &nbsp;2004; 28: 1333&ndash;1337</div>\r\n	<div>\r\n		Wilborn, C., Beckham, J., Campbell, B., Harvey, T., Galbreath, M., La Bounty, P., Nassar, E., Wismann, J., Kreider, R. 2005. Obesity: Prevalence, Theories, Medical Consequences, Management, and Research Directions. J of the International Society of Sports Nutrition. September. &nbsp;</div>\r\n	<div>\r\n		Zalilah, M.S., Khor, G.L., Mirnalini, K., Norimah, A.K., Ang, M. 2006. Dietary intake, physical activity and energy expenditure of Malaysian adolescents. Singapore Med J 2006; 47(6):491-498</div>\r\n	<div>\r\n		&nbsp;</div>\r\n</div>\r\n', 1),
(1036, 1029, NULL, NULL),
(1040, 1033, '<div>\r\n	Palmaris longus (PL) is a muscle of the flexor compartment of the forearm. The muscle originates from the medial epicondyle of the humerus and it is attached to the distal half of the flexor retinaculum and apex of the palmar aponeurosis (Parson 2009). The tendon crosses the flexor retinaculum to diverge into four ligamentous fibrous slips for all the digits except the thumb (Williams et al. 1995). These four slips form the palmar aponeurosis. Thus, there is genuine reason to believe that the degenerated tendon of PL exists as palmar aponeurosis. The PL is innervated by a branch from the median nerve.</div>\r\n<div>\r\n	Considering the embryological aspect, the existence of any anomalous muscle may be explained with regard to the abnormal persistence of any undifferentiated group of mesenchymal cells involved in the formation of upper limb muscles (Soldado-Carrera et al. 2000). Due to evolution, there may be cessation or persistence of muscle mass. It is noteworthy to perform future research studies and relate the anomalous PL to any particular physical profession which involves rigorous use of flexor muscles of the forearm.</div>\r\n<div>\r\n	The PL has been reported to be absent unilaterally or bilaterally in many individuals (Stack 1973). There wre interesting cases of PL being reversed, duplicated, bifid or being hypertrophied (Murabit et al. 2013). Additional slips may be present. Research studies showed that Egyptians had the highest rate of the absence of the PL muscle (Raouf et al. 2013). Interestingly, the African Americans and Asians were reported to have less prevalence of absence of PL compared to the Caucasians (Soltani et al. 2012).</div>\r\n<div>\r\n	Researchers carried out studies to ascertain the clinical implications. It was observed in both sexes, the grip strength of the hand was not affected in the absence of the PL but the pinch strength of the fourth and fifth digit decreased (Cetin et al. 2013). The PL may be important as a suitable graft and even surgeons consider it to be a useful anatomical landmark for hand surgeries. Anomalous PL in the carpal tunnel may even produce carpal tunnel syndrome or compartment syndrome.</div>\r\n', 1);
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(1042, 1035, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Honey is a natural sweet substance produced by honey bees from the nectar of blossomed flowers which is called blossom (floral) or nectar honey and can either be unifloral (nectar from same flower) or mulifloral (nectar of various types of flowers). It can also be found from the secretions of living parts of plants or excretions of plant sucking insects on the living parts of plants which is called honeydew (non floral) honey. The collected nectar and honeydew by the honey bees are transformed and combined with specific substances from the bees, and stored in the honey comb to ripen and mature (Codex Alimentarius Commission 2001).</div>\r\n<div>\r\n	Honey production and its use have a long and varied history. It has been traditionally used as food and medicine products since the earliest times by primeval humans as well as the Ancient people of Egypt, China, India, Greece and Rome. The first written reference about honey was on a clay tablet of the Sumerians in the Euphrates valley dating back to 2100-2000 BC, which mentioned the use of honey as a drug and an ointment (Crane 1975).</div>\r\n<div>\r\n	In the year 200 AD, it was mentioned in a written form in ancient Chinese Medicine Shennong, in which many prescriptions and medical indications contain honey (Bogdanov 2010a). Likewise, in ancient India, Ayurvedic medicine (a Hindu system of traditional medicine native to India and a form of alternative medicine) uses honey for many purposes.</div>\r\n<div>\r\n	According to the bible, King Solomon has said: &ldquo;Eat honey my son, because it is good&rdquo; (Old Testament, proverb 24:13). In Islam, there is an entire surah in the Al-Qur&rsquo;an called Al-Nahl (the bee) that narrates the goodness of honey: &ldquo;And your Lord inspired to the bee, take for yourself among the mountains, houses, and among the trees and [in] that which they construct. Then eat from all the fruits and follow the ways of your Lord laid down [for you]. There emerges from their bellies a drink, varying in colors, in which there is healing for people. Indeed in that is sign for a people who give thought&rdquo; (Al-Qur&rsquo;an, Al-Nahl 16:68-69) and also Allah says that &ldquo;In paradise will be rivers of pure honey&rdquo; (Al-Qur&rsquo;an, Muhammad 47:15). Prophet Muhammad (peace be upon him) strongly recommended honey for the healing purposes and commented on its good values 1400 years ago (Sahih Bukhari).</div>\r\n<div>\r\n	In the past decade, the benefits of honey become more focused on its medicinal properties such as antibacterial, antioxidant, anti-inflammatory and anticancer properties (Al-Mamary et al. 2002; Ghashm et al. 2010; Mohd Nasir et al. 2010; Wen et al. 2012). An alternative medicine branch called &ldquo;Apitherapy&rdquo; has developed offering treatments based on honey and other bee products against many diseases (Bogdanov 2009a).</div>\r\n<div>\r\n	In this review, we highlighted the components present in honey, its therapeutic properties beneficial to human health as well as its nutritional values based on scientific evidences.</div>\r\n<div>\r\n	MODE OF PROCESSING</div>\r\n<div>\r\n	Honey can be categorized regarding the mode of processing given below (Joshi 2008):</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&bull; Squeezed honey: a traditional method of honey extraction, which involves squeezing the honey combs.</div>\r\n<div>\r\n	&bull; Drained honey: honey is obtained by draining decapped and broodless combs.</div>\r\n<div>\r\n	&bull; Extracted honey: honey is obtained by centrifuging decapped honey combs which is mainly produced by beekeepers who manage bees in moveable comb hives.</div>\r\n<div>\r\n	&bull; Pasteurized honey: honey that has been heated in a pasteurization process (71.7˚C or higher). Pasteurization reduces the moisture level, destroys yeast cells and liquefies any microcrystals in the honey. However, excessive heat exposure also results in product deterioration, as it increases the level of hydroxymethylfurfural (HMF) and reduces enzyme activity (e.g. diastase). The heat also affects appearance (darkens the natural honey color), taste and fragrance (Manley 1985).</div>\r\n<div>\r\n	CONSISTENCY AND APPEARANCE</div>\r\n<div>\r\n	Honey can be classified also according to its consistency and appearance (Joshi 2008):</div>\r\n<div>\r\n	&bull; Liquid honey: the honey is thinner or thicker in consistency and also free of any visible crystals.</div>\r\n<div>\r\n	&bull; Crystallized honey: honey is completely granular or solidified; it is also called &ldquo;granulated honey&rdquo;.</div>\r\n<div>\r\n	&bull; Partially crystallized honey: a mixture of liquid and crystallized honey.</div>\r\n<div>\r\n	COLOR OF HONEY</div>\r\n<div>\r\n	The color of honey can be categorized into seven categories with Pfund color scale of 0 to more than 114 mm, as shown in Table 1 (The National Honey Board 2010). The color of honey partly reflects the content of pigments with antioxidant properties such as carotenoids and flavonoids (Khalil et al. 2011). In addition, the color intensity of honey may be considered as a good indication of its antioxidant capacity (Pilijac-Zegarac et al. 2009).</div>\r\n<div>\r\n	COMPOSITION OF HONEY</div>\r\n<div>\r\n	Honey is a supersaturated solution of sugars, mainly composed of fructose and glucose, containing also other important compounds such as minerals, proteins, enzymes, free amino acids, vitamins and many phenolic compounds (Alvarez-Suarez et al. 2010) as well as some organic acids and volatile substances (Perez et al. 2002; Mato et al. 2003). The composition of honey is summarised in Table 2 (The National Honey Board 2010). The composition of honey is rather variable and primarily depends on the botanical origin and the floral source (Persano-Oddo &amp; Piro 2004). However, certain external factors also play a role such as seasonal, environmental, processing, handling, storage, and climatic conditions (Lachman et al. 2010).</div>\r\n<div>\r\n	CARBOHYDRATES</div>\r\n<div>\r\n	Honey is mainly made up of carbohydrates (82.3%) which is a highly complex mixture of sugars. The major sugars in honey are monosaccharides consisting of fructose (38%) and glucose (31%) (Lawal et al. 2009; Alvarez-Suarez et al. 2010) which are responsible for most of the physical and nutritional charactristics of honey (Sato &amp; Miyata 2000). Some of the disaccharides have also been identified in honey such as sucrose, which is composed of fructose and glucose linked together, maltose, kojibiose, turanose, isomaltose, and maltulose, which comprise less than 8% of the composition of honey. Honey also contains trisaccharides such as melezitose and raffinose (Cotte et al. 2004; The National Honey Board 2010).</div>\r\n<div>\r\n	MOISTURE</div>\r\n<div>\r\n	The moisture in honey or water content is the most important component and related to honey quality especially concerning the risk of spoilage due to fermentation and granulation or crystallization during storage (Finola et al. 2007). In general, moisture content in ripe honey is less than 20% while under very humid or tropical conditions the moisture can be more than 20% (Bogdanov 2010b) but only honeys with less than 18% moisture content can be stored with little or no risk of fermentation.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	PROTEINS, ENZYMES AND AMINO ACIDS</div>\r\n<div>\r\n	Honey naturally contains small amount of enzymes that are introduced into honey by the bees during various phases of the honey manufacturing process. The three main honey enzymes are amylase, which decomposes starch or glycogen into smaller sugar units dextrin and maltose, invertase which converts sucrose to fructose and glucose, and glucose oxidase which produces hydrogen peroxide and gluconic acid from glucose in the presence of water (Bansal et al. 2005; Bogdanov et al. 2008). Other enzymes present in lesser amounts are catalase and acid phosphatase (Weston 2000; The National Honey Board 2010).</div>\r\n<div>\r\n	The amino acids identified in honey from different botanical and geographical origin are: glutamic acid, aspartic acid, glutamine, histidine, glycine, threonine, &beta;-alanine, arginine, &alpha;-alanine, &gamma;-aminobutyric acid, proline, tyrosine, valine, methionine, cysteine, isoleucine, leucine, tryptophan, phenylalanine, ornithine and lysine (Iglesias et al. 2004; Perez et al. 2007).</div>\r\n<div>\r\n	VITAMINS AND MINERALS</div>\r\n<div>\r\n	Honey contains varying amounts of vitamins, minerals and trace elements. The vitamins content in honey is low. However, vitamins such as phyllochinone (K), thiamin (B1), riboflavin (B2), niacin (B5) and pyridoxine (B6) as well as vitamins A, E and C have been reported to be present in honey (Al-Waili 2003; Bogdanov et al. 2008). The minerals content of honey depends mainly on the botanical and geographical origin of honey (Bogdanov et al. 2007). Light color blossom honeys have a lower mineral content than dark color honeys (Bogdanov 2009b). The minerals commonly found in honey are potassium, aluminium, cadmium, barium, nickel, chromium, cobalt, antimony, calcium, iron, zinc, mangnesium, copper, sodium, manganese and selenium (Stocker et al. 2005; Bogdanov 2009b).</div>\r\n<div>\r\n	VOLATILE SUBSTANCES</div>\r\n<div>\r\n	The main volatile compounds in honey have their origins in different chemical families such as alcohols, ketones, aldehydes, acids, esters and terpenes and are present in honey at very low concentrations. The volatile substances mainly derived from the nectar sources give distinctive flavours and aroma to honey (Zhou et al. 2002; Cuevas-Glory et al. 2007).</div>\r\n<div>\r\n	POLYPHENOLIC COMPOUNDS</div>\r\n<div>\r\n	Polyphenolics are important group of organic compounds responsible for the appearance of honey and its antioxidant capacity (Tomas-Barberan et al. 2001). The polyphenols present in honeys are flavonoids such as hesperetin, kaempferol, quercetin and chrysin, and phenolic acids such as p-coumaric, abscisic, ellagic, gallic and ferulic acids (Yaoa et al. 2005; Kenjeric et al. 2007). Our laboratory found the following polyphenols to be prominent in Malaysian Gelam honey as evaluated by HPLC method: caffeic acid, chlorogenic acid, p-coumaric, quercetin and hesperitin (Hussein et al. 2011).</div>\r\n<div>\r\n	HYDROXYMETHYLFURFURAL (HMF)</div>\r\n<div>\r\n	Hydroxymethylfurfural (HMF) or 5-hydroxymethyl-2-furaldehyde, is a heterocyclic aldehyde (Figure 1) (Bogdanov 2009b) which forms as a result of hexoses degradation in an acidic environment (Semkiw et al. 2010). The presence of simple sugars such as glucose and fructose with many acids in honey is a favorable condition for the production of this substance (Khalil et al. 2010).</div>\r\n<div>\r\n	HMF is almost absent or very low in fresh honey and its concentration increases as a result of heating processes, long-term storage or stored in non-adequate conditions, use of metallic containers and adulterated with invert syrup. HMF measurement is used to evaluate the freshness and quality of honey (Zappala et al. 2005; Ajlouni &amp; Sujirapinyokul 2010). The Codex Alimentarius (2001) and International Honey Commission (2002) set the maximum concentration of HMF to 40 mg/kg for honey from non-tropical region and 80 mg/kg for honey from tropical regions. Honey is thus best consumed within six months of purchase to reduce the production of HMF.</div>\r\n<div>\r\n	NUTRITIONAL BENEFITS OF HONEY</div>\r\n<div>\r\n	Honey is one of the oldest and best-loved sweetening agents for foods from olden days until now (Al-Qassemi &amp; Robinson 2003). It is an important source of carbohydrates and the only widely available sweetener which provides strength and energy to our body (Benefits of Honey 2012). The carbohydrates in honey are easily digested and quickly transported into the blood to be utilized for energy requirements by the human body. The glucose in honey is absorbed by the body quickly and gives an immediate energy boost, while the fructose is absorbed more slowly providing sustained energy. It is known that honey has also been found to keep levels of blood sugar fairly constant compared to other types of sugar. Thus, honey is particularly recommended for children and sportsmen because it can help to improve on the efficiency of the energy system of the elderly and invalids (Alvarez-Saurez et al. 2010; Benefits of Honey 2012).</div>\r\n<div>\r\n	THERAPEUTIC PROPERTIES OF HONEY</div>\r\n<div>\r\n	Apitherapy (the medical use of honey bee products) has recently become the focus of attention as a form of preventive medicine for treating certian conditions and diseases, as well as promoting overall health and well being. Table 3 summarizes data collected from animal and human studies on the health beneficial effect of honey. It has been reported to be effective as an antioxidant, in gastrointestinal disorders, in the healing of wounds and burns, and as an antimicrobial and anti-tumour agents. However, because some of these diseases are a consequence of oxidative damage, it seems that part of the therapeutic properties of honey is due to its antioxidant capacity (Jaganathan &amp; Mandal 2009; Ferreira et al. 2009)</div>\r\n<div>\r\n	ANTIOXIDANT ACTIVITY</div>\r\n<div>\r\n	Gelam honey has antioxidative and radical scavenging activities, which are mainly attributed to its phenolic contents (Hussein et al. 2011; Khalil et al. 2012). Erujewa et al. showed that Tualang honey supplementation reduces considerably elevated systolic blood pressure via amelioration of oxidative stress in the kidney of spontaneously hypertensive rats, SHR (Erujewa et al. 2012). The gene expression of Nrf2, a potential renoprotective transcription factor, which was markedly reduced or impaired in the kidney of SHR was upregulated by honey supplementation (Erujewa et al. 2012).</div>\r\n<div>\r\n	PROTECTIVE EFFECT IN GASTROINTESTINE</div>\r\n<div>\r\n	Infections of the intestinal tract are common throughout the world and affect people of all ages. The use of honey for prevention and treatments of gastrointestinal disorders such as peptic ulcers, gastritis and gastroenteritis has been reported in various publications around the world (Bogdanov et al. 2008). Honey has antimicrobial activity against bacteria that can cause diarrhea especially the ones that are caused by E. coli (Adebolu 2005) and against many enteropathogenic organisms including those of the Salmonella and Shigella species (Molan 2001a).</div>\r\n<div>\r\n	Helicobacter pylori (H. pylori) infection is probably one of the most common bacterial infections worldwide and complications resulting from this infection caused gastritis, gastric and duodenal ulcers (Sherif et al. 2004; Tiwari et al. 2005). Honey derived remedies comprise a potential source of new compounds that may be useful in the management of H. pylori infections (Manyi-Loh et al. 2010). A study carried out by Ndip et al. (2007) on four selected honeys (Mountain, Manuka, Capillano and Eco) was found to exhibit antibacterial activity against H. pylori associated gastrointestinal disease.</div>\r\n<div>\r\n	Honey was also shown to have a dose-dependent effect protecting the stomach from ulceration caused by indomethacin (an aspirin-type anti-inflammatory drug) and alcohol in rats experimentally induced with peptic ulcer (Ali &amp; Al-Swayeh 1997; Gharzouli et al. 2002).</div>\r\n<div>\r\n	CARDIOVASCULAR HEALTH</div>\r\n<div>\r\n	Yaghoobi et al. (2008) studied the effect of honey in normal and overweight persons with higher risk for cardiovascular disease. The normal persons received 70 g of sucrose while cardiovascular disease patients received 70 g of honey, for 30 days. The effect of natural honey was investigated on total cholesterol, LDL-C, HDL-C, triacylglyceride, fasting blood glucose (FBG) and body weight in overweight individuals. The results showed that consumption of natural honey reduces cardiovascular risk factors, by modulating the plasma levels of total cholesterol, low-density lipoprotein cholesterol (LDL-C), high-density lipoprotein cholesterol (HDL-C), triacylglyceride and homocysteine. It is highly likely that the antioxidant polyphenols in honey such as quercetin, acacetin, caffeic acid, phenethyl ester, kaempferol and galangin are the components with promising pharmaceutical action in the treatment of cardiovascular diseases (Khalil &amp; Sulaiman 2010).</div>\r\n<div>\r\n	WOUNDS AND BURNS HEALING</div>\r\n<div>\r\n	The healing properties of honey have been known since time immemorial and have recently gained recognition from the scientific community (Molan 2000). Honey has been used for the treatment of chronic wounds, burns, diabetic foot ulcers and many other ailments (Visavadia et al. 2008; Simon et al. 2009). Honey accelerates wound healing whether applied topically or administered systemically. An infected wound will not heal unless bacteria are eliminated because bacteria stimulate the inflammatory response which can prevent or slow the process of wound healing. Molan (2002) reported that honey has an inhibitory effect on approximately 60 species of bacteria such as aerobes and anaerobes as well as gram-positive and gram-negative bacteria. Honey creates a moist environment without the risk of bacterial growth and with no adverse effects to slow the healing process. Honey has high viscosity which provides a protective barrier thus preventing cross-infection (Molan 2006; Khoo et al. 2010).</div>\r\n<div>\r\n	Therapeutic effects of honey in burn treatment have been reported to cause rapid healing of wounds with less scarring (Yusof et al. 2007). Molan (2001a) found that the application of New Zealand Manuka honey accelerated healing in acute wounds, chronic ulcers, burns and infected wounds. Zohdi et al. (2004) reported that topical application of Malaysian Gelam honey in the form of hydro gel dressings was effective in accelerating wound healing due to burn in rats (Zohdi et al. 2012). In another study, Aljadi et al. (2000) found that combined Gelam honey treatment (topical and oral) offers a distinct advantage to wound healing and may be a useful adjuvant in wound management. Malaysian Tualang honey was able to control the infection from Pseudomonas aeruginosa and accelerates contraction of wound due to burn (Khoo et al. 2010). Zaharil et al. (2011) found that Tualang honey impregnated dressings were as effective as silver impregnated hydrofibre dressings in terms of dressing properties, promotion of wound healing and inflammatory reaction for full thickness wounds in rats.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ANTIMICROBIAL AND ANTIVIRAL ACTIVITIES</div>\r\n<div>\r\n	The major contributor to the antimicrobial activity of honey is hydrogen peroxide (Temaru et al. 2007) which is formed from the oxidation of glucose by glucose oxidase to gluconic acid and hydrogen peroxide when honey is diluted (Bang et al. 2003; Iurlina &amp; Fritz 2005). The varying antimicrobial activity in different types of honey is because of the different concentrations of hydrogen peroxide (Molan 1999). The antimicrobial activity of honey is also attributed by some physical factors such as acidity (low pH) and osmolarity (Hamouda &amp; Abouwarda 2011). The high osmolarity of honey is due to the high content of sugar (over 85% of honey) and these sugars have high affinity for water molecules leaving little or no water to support the growth of different microorganisms such as bacteria and yeast. Consequently, the microorganisms become dehydrated and eventually die (Hamouda &amp; Abouwarda 2011). Honey is acidic with a pH range of 3.2 to 4.5 which is low enough to inhibit the presence and growth of many pathogens (Allen et al. 1991).</div>\r\n<div>\r\n	Honey has a broad-spectrum antimicrobial activity on gram-negative and gram-positive bacteria (Agbaje et al. 2006). Several studies have revealed that honey is effective against Methicillin resistant Staphylococcus aureus (MRSA), &beta;-hemolytic Streptococci and Vancomycin-resistant Enterococci (VRE) (Allen et al. 2000; Kingsley 2001). Due to its antibacterial activity, honey has carioprotective effect by inhibiting the growth of bacteria causing caries (Steinberg et al. 1996; Sela et al. 1998; Molan 2001b). It was reported that Manuka honey has a positive effect against dental plaque development and gingivitis (English et al. 2004) making it a useful substitute of refined sugar in the manufacture of candy (Molan 2001b).</div>\r\n<div>\r\n	Honey can also inhibit the growth of a wide range of fungi, protozoa and viruses (Blair &amp; Carter 2005). It was shown to have inhibitory effect on the Rubella virus (Zeina et al. 1996) and Leishmania parasite (Zeina et al. 1997) as well as fungi such as Candida spp. and Trichosporon spp. (Koc et al. 2009).</div>\r\n<div>\r\n	ANTI-TUMOR ACTIVITY</div>\r\n<div>\r\n	Many studies have shown that high polyphenols in honey has been attributed for its anti-tumor property (Russo et al. 2004; Jaganathan &amp; Mandal 2009a). Jaganathan &amp; Mandal (2009b) illustrated the ability of the honey to induce apoptosis in human colorectal cancer cells attributed by the high phenolics and tryptophan contents. Additionally, Malaysian Tualang honey was found to have significant anticancer activity against human breast cancer (MCF-7) and cervical cancer (HeLa) cell lines (Fauzi et al. 2011) by reducing mitochondrial membrane potential and activating caspase-3/7 and -9, leading to apoptosis. Ghashm et al. (2010) showed that Tualang honey induces apoptosis in oral squamous cell carcinomas (OSCC) and human osteosarcoma (HOS). Wen et al. (2012) reported that Malaysian Gelam and Nenas honeys were capable of suppressing the growth of HT 29 colon cancer cells by inducing apoptosis and suppressing inflammation. Swellam et al. (2003) showed that honey inhibited the growth of bladder cancer cell lines in vitro as well as in vivo when administered intralesionally or orally in the bladder cancer implantation models.</div>\r\n<div>\r\n	ANTI-INFLAMMATORY ACTIVITY</div>\r\n<div>\r\n	Inflammation is the response of vascularized living tissue to local injury and plays an important role in various diseases such as rheumatoid arthritis, atherosclerosis and asthma, which all show a high prevalence globally (Tran et al. 2009; Mueller et al. 2010). During an inflammatory response, several pro-inflammatory mediators are released including interleukins (IL-1, IL-6, IL-2 and IL-8), tumor necrosis factor (TNF) and interferon (IFN-&gamma;) as well as cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS) which are suppressed by anti-inflammatory cytokines such as IL-4, IL-10, IL-13, IFN-&alpha; and transforming growth factor (TGF-&beta;), which therefore help to balance the inflammatory response (Hanada &amp; Yoshimura 2002; Goldstein et al. 2006; Mueller et al. 2010). Thus, inhibition of the overproduction of inflammatory mediators, especially pro-inflammatory cytokines (IL-1&beta;, IL-6 and TNF-&alpha;), may prevent or suppress a variety of inflammatory diseases (Kim et al. 2003).</div>\r\n<div>\r\n	Honey has been reported to have anti-inflammatory effect in in vitro and in vivo studies. Kassim et al. (2010a) found that Gelam honey extract inhibited the level of nitric oxide (NO) in LPS-stimulated macrophage RAW264.7 cells. Honey and its extracts have anti-inflammatory effects by reducing the inflammatory mediators NO and PGE2 in rat paw tissues (Kassim et al 2010b). It was also shown to decrease inflammation in an experimental model of inflammatory bowel disease in rats (Prakash et al. 2008). Owayele et al. (2011) reported that honey can inhibit the NO release in carrageenan-induced inflammation in rats.</div>\r\n<div>\r\n	Our research group has demonstrated that Malaysian Gelam honey has a potent in vivo anti-inflammatory effect by inhibiting the production of pro-inflammatory mediators NO, PGE2, TNF-&alpha;&alpha;and IL-6 in plasma of carrageenan-induced paw oedema inflammation.</div>\r\n<div>\r\n	REPRODUCTIVE HEALTH</div>\r\n<div>\r\n	There is a traditional belief that honey may enhance fertility and vitality as well as improve male reproductive health. It had been reported that daily treatment of 5% Palestinian honey for 20 days increased spermatogenesis in adult rats (Abdul-Ghani et al. 2008). Abdelhafiz &amp; Muhamad (2008) observed that diluted Egyptian honey and royal jelly had an enhancing effect on sperm motility, particularly in subnormal samples (in vitro study).</div>\r\n<div>\r\n	Mahaneem et al. (2007, 2011) reported that Tualang honey (1.2 g/kg of body weight) significantly increased the percentages of rats achieving intromission and ejaculation as well as increased mating and fertility indexes of male rats exposed to Cigarette Smoke (CS) (Mohamed et al. 2012).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Their result suggests that honey has a protective effect against CS-induced impaired sexual behavior and fertility in male rats. Likewise, Asiyah et al. (2011) reported that Malaysian Gelam honey (1.0 ml/100g of body weight) was potentially useful in increasing the fertility of juvenile male rats by increasing sperm motility and number of morphologically normal sperm compared with male rats injected with nicotine.</div>\r\n<div>\r\n	A study by Zaid et al. (2010) on the effects of Tualang honey on female reproductive organs (uterus and vagina) and tibia bone in menopause rats (ovariectomised) showed that Tualang honey has a beneficial effect on menopausal rats by preventing uterine atrophy, increased bone density and suppression of body weight increase. Thus, these findings may suggest honey could be an alternative to hormone replacement therapy (HRT).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Honey is one of the oldest and best sweetening agents for foods over the centuries. It is made up of a vast amount of different compounds that can be of nutritional and health benefits. The main nutrition and health-relevant components are the carbohydrates, especially fructose and glucose, which make it an excellent energy source for human. Honey also contains a great number of other constituents in small and trace amounts including proteins, enzymes, vitamins, minerals and phenolics compounds, producing numerous nutritional and biological effects such as: wound healing, antimicrobial, antiviral, antioxidant, anti-inflammatory and antitumor activities. Thus, the use of honey either as a food or an alternative medicine in the treatment of different disorders is safe, beneficial and free from side effects.</div>\r\n<div>\r\n	ACKNOWLEDGMENT</div>\r\n<div>\r\n	This study was supported by grants from the Fundamental Research Grant Scheme (FRGS), (FF-03-FRGS0039-2010) UKM and the National University of Malaysia Fundamental Fund (FF-156-2010).</div>\r\n', 1),
(1044, 1037, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Stroke is one of the main causes of adult disability and has been the top four leading causes of death in developing countries. It is expected that the prevalence of stroke would be doubled in the next decade, corresponding to the increase in life expectancy and in lifestyle related illnesses namely hypertension, hyperlipidaemia and obesity. In Malaysia alone, stroke ranked as the fifth highest cause of death in 2009 with a mortality rate of Malaysia8.43 per 100 000 population (MOH 2009).</div>\r\n<div>\r\n	Management of stroke is through a multidisciplinary approach incorporating acute stroke units, seamless transfer of care from hospital to home, structured rehabilitation and comprehensive long term care to address post stroke complications and also management of secondary risk factors (NICE 2008; Yong &amp; Foster 2007; Langhorne et al. 2011). It is acknowledged that approximately two thirds of stroke survivors would survive the acute episode of stroke but many of these survivors may be living with various degrees of disabilities and dependent for the rest of their life (Internet Stroke Center 2013). Fragmented community services post transfer of care was identified as one of the barriers for optimum recovery; together with other issues related to the elderly population such as exposure to the risk of falls, dementia and safety (Young 2001).</div>\r\n<div>\r\n	Over the last two decades, stroke rehabilitation has been acknowledged as an integral part of stroke recovery, with specific restorative and repetitive learning processes tailored to individual disabilities; this is the key to improving the survivor&rsquo;s physical function in performing activities of daily living and mobility over time (Langhorne et al. 2011). Nonetheless, the degree of recovery depends on several factors, namely the patient&rsquo;s age, functional independence, support system and premorbid condition (Langhorne et al. 2009; Jorgensen et al. 1995; Musicco et al. 2003). Cardiovascular risk factors, notably diabetes, were also identified to influence the recurrence and rate of recovery. For example, a study done by Hamidon and Raymond found that diabetes is an independent risk factor influencing post stroke complications, which may result in poorer outcome after strokes as these patients tend to be more prone to infection and already had pre existing end organ damages secondary to diabetes (Hamidon &amp; Raymond 2003).</div>\r\n<div>\r\n	The management of these risk factors requires long term commitment by medical personnel extending beyond in-hospital care. Guidelines have advocated the need of structured transfer of care from hospital to primary care as an important element for long term care and control (Langhorne et al. 2005). This study looked at early functional recovery and its associated factors in post ischemic stroke patients after they have been discharged from a tertiary hospital back to the community. It is hoped that the findings of this trial can be used to initiate a better transfer of care plan from the hospital to the community doctors by incorporating multidisciplinary team collaboration between in hospital physicians, primary care doctors, rehabilitation personnel and other support team members.</div>\r\n<div>\r\n	METHODOLOGY</div>\r\n<div>\r\n	This study was approved by the Medical Research and Ethics Committee of the Universiti Kebangsaan Malaysia (UKM).</div>\r\n<div>\r\n	DESIGN</div>\r\n<div>\r\n	A prospective study looking at 37 newly diagnosed stroke patients admitted to Universiti Kebangsaan Malaysia Medical Centre (UKMMC) from December 2010 to June 2011 with a primary diagnosis of stroke based on the WHO definition of stroke (Hatano 1976). Those who were comatose or somnolent and had substantial physical and cognitive impairment as a result of previous neurological and mental illness were excluded from the study. Each patient was seen at admission. Those who consented were assessed in terms of their functional status using the Modified Barthel Index (MBI) at baseline and at three months.</div>\r\n<div>\r\n	The MBI was developed by Shah et al. (1989) derived from the original 1965 Barthel&rsquo;s Index by Mahony and Barthel, a 10-question self reported questionnaire that measures physical disability based on ability to perform basic activities of daily living (ADL) (Mahoney &amp; Barthel 1965). It is the most common instrument used to measure functional disability in stroke rehabilitation studies (Sangha et al. 2005). A score of less than 50 was taken as &ldquo;dependent&ldquo; since subjects below this score were unlikely to be able to perform any basic activities of daily living (ADL) such as showering and mobilising without being assisted. The demographic and clinical information of the stroke patients were also assessed at baseline.</div>\r\n<div>\r\n	All stroke patients in this study were followed up as standard care for any stroke patient admitted to the hospital. At three months post stroke, patients were reassessed for functional status using the MBI and also for depression using the Patient Health Questionnaire 9-item depression scale (PHQ-9). This 9-item self-administered questionnaire was developed by Spitzer (Spitzer et al. 1999). It is based on the 9 DSM-IV symptoms of depression, which facilitates its use as a depression diagnostic tool as well as a screening instrument. It has been validated for use in primary care and stroke patients (Dietrich et al. 2003).</div>\r\n<div>\r\n	Other outcomes such as mortality and placement post hospital discharge, were also collected. Those who were not contactable physically at the respective outpatient clinics were interviewed by phone. A defaulter was labelled for patients who failed to be contacted through the clinic appointments and by phone.</div>\r\n<div>\r\n	STATISTICAL ANALYSIS</div>\r\n<div>\r\n	Data was analysed using the Statistical Package for the Social Sciences (SPSS) version 13. Descriptive statistics were used to described demographic and clinical profiles. Functional recovery comparing baseline and three months MBI scores were analysed using the Wilcoxon signed Rank Test. Bivariate association between the demographic and clinical profiles were analysed using the Student&rsquo;s T Test and ANOVA. A p-value of less than 0.05 and confidence interval of 95% were considered as statistically significant.</div>\r\n<div>\r\n	RESULT AND ANALYSIS</div>\r\n<div>\r\n	A total of 46 ischaemic stroke patients were recruited during the 6-month study period. At the end of the three months observational period, seven patients (15.2%) had died and two patients (4.3%) were lost to follow up. A total of 37 patients (84.7%) were still alive and continued in the study. A significant proportion of the patients were living at home (69.6%) with their family members, 6.5% readmitted to hospitals for various reasons and 4.3% were living in nursing homes. The demographic and clinical profiles are as presented in Table 1 and Table 2 respectively.</div>\r\n<div>\r\n	Looking into the clinical profile of the stroke patients, 30.4% of the subjects had recurrent strokes and 6.5% had had TIA in the past. Hypertension (89.1%) was the commonest risk factor for stroke patients in this study followed by dyslipidaemia (65.2%) and diabetes mellitus (63.0%).</div>\r\n<div>\r\n	The functional status of the patients at the end of the three months observational period demonstrated a significant improvement in terms of the MBI score (Table 3). The PHQ-9 score, which was also assessed at the end of the observed period demonstrated that 21.6% of patients had some form of depression.</div>\r\n<div>\r\n	Bivariate analysis of functional recovery and PHQ-9 demonstrated that lower functional recovery was significantly found among depressed patients (p&lt;0.026). Among other variables, only patients with recurrent stroke were found to have had lower functional recovery (p&lt;0.05).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This was a prospective study that observed the functional outcome of 37 post stroke patients after being discharged from a tertiary hospital care in Malaysia. At baseline 78.4% of patients were found to be dependent and 21.6% were independent. At three months post stroke, only 18.9% of the patients were still dependent with functional recovery from baseline to three months noted to be significant (p&lt;0.01) with a mean difference in Barthel&rsquo;s Index of 45.2 (SD 27.0). These findings showed that the speed and extent of recovery are important in planning the care and the need for rehabilitation during the recovery period.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Local studies looking at functional outcomes showed that the number of stroke patients who were still dependent on their carer within the three to six months follow up varied from 18.9% to 40% which is comparable to the present findings (Jaya et al. 2002; Rameezan &amp; Zaliha 2005).</div>\r\n<div>\r\n	However, most outcome studies on stroke varied in their methodology and used different tools to access function and used different cut off points to define dependency level. Therefore, it was not possible to compare outcomes between these studies. In spite of this, this study also demonstrated that a significant number of stroke survivors were still dependent on their carers well after they have been discharged from hospital; thus the need for good outpatient medical support network for a structured and continued medical care.</div>\r\n<div>\r\n	Our stroke patients were mainly elderly with multiple risk factors with a mean age of 67.2&plusmn;11 years old. This is expected as most strokes occur in the elderly and is comparable to other studies conducted in Asian and Western countries (Venketasubramanian 1999; Rothwell et al. 2004). With the increasing life span of the world&rsquo;s population, the mean age for stroke is expected to rise unless effective health promotion and risk prevention efforts are made in the community, especially by the primary health care services (Krishnamoorthy 2007).</div>\r\n<div>\r\n	The top five risk factors identified in this study were all modifiable risk factors through better lifestyle choices and better preventive measures from the health care system in the community. Similarly, hypertension was identified to be the most common risk factor for stroke locally and despite the high prevalence of hypertension in the world, the level of awareness, treatment and control of hypertension in the community remain low especially in developing countries including Malaysia (Internet Stroke Center 2013; Jaya et al. 2002; Rampal et al. 2008). Dyslipidaemia and diabetes were the second and third commonest risk factors for stroke in this study. These two lifestyle diseases are also among the top risk factors associated with stroke in the East Asian region including Australia (Burke &amp; Venketasubramanian 2006; Ong &amp; Raymond 2002). These three risk factors are sufficient for the diagnosis of metabolic syndrome, which is strongly associated with higher cardiovascular consequences and was also found to have more negative outcomes from stroke (Hamidon &amp; Raymond 2003; Oh et al. 2013). Despite this, our study did not find any significant association of these factors with functional recovery after stroke.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	One third or 30.4% of the subjects recruited in this study had a previous stroke and another 28.3% had ischemic heart disease in the past, which may suggest the lack of attention given to secondary prevention strategies. Previous studies had shown that, patients with previous stroke had slower functional recovery when compared to patients with a first ever stroke as many of them suffered some form of residual disability hampering the recovery process (Kwakkel et al. 1996; Hankey et al. 2002; Jorgensen et al. 1997). This study did not directly assess the association between functional recovery and number of stroke due to the small sample size (Kwakkel et al. 1996).</div>\r\n<div>\r\n	Nearly 21.6% of the patients were found to have depression at three months. This is not uncommon, as post stroke depression (PSD) has been reported to occur in approximately 10% to 50% of all stroke patients after a few months post stroke (Glamcevski &amp; Pierson 2005; Sulaiman et al. 2002). Local studies reported that the prevalence of depression ranges from 36% at one to two months to 66% at three to six months post stroke (Glamcevski &amp; Pierson 2005; Sulaiman et al. 2002). PSD has been associated with increased mortality, disability, length of hospital stay and reduced health-related quality of life (Townend et al. 2007). Similarly, our study also found that patients with depression at three months had a significantly lower functional recovery from their stroke with a mean difference of 22.79 (SD=2.88, 42.69) in the PHQ-9 score when compared to the non depressed stroke patients. Depression is a very debilitating mental problem that may result in social isolation causing patients to be unable to perform certain activities that can affect the rehabilitation process and hinder recovery.</div>\r\n<div>\r\n	Depression not only delays functional recovery, but on the other hand, those with delayed functional recovery may also succumb to depression (Glamcevski &amp; Pierson 2005; Townend et al. 2007). Nevertheless, Townend et al. (2007) reported that depression may also occur even with the improvement of functional status (Townend et al. 2007). It was suggested that rather than the rate of recovery, the initial impact of disability brought by the stroke insult could pre determine the onset of depression. Despite the detrimental effect of depression, PSD is often under-recognised and under-treated in the community even within non-stroke patients (MOH 2006). This may stem from the social stigma associated with the condition or failure to recognise the depressive symptoms as significant by the sufferers or by the treating primary care doctors (Lotfi et al. 2010).</div>\r\n<div>\r\n	For primary care physicians, this study demonstrates the need to be aware of the caveats of managing stroke patients after being discharged to the community. These include mandatory screening for cardiovascular risk factors and depression, documentation of functional status and provision of rehabilitation. The management should be coordinated between hospital and primary care services. The coordination of care is important, as management of stroke care post discharge is complex, involving managing risk factors, preventing recurrent stroke and further rehabilitation. It is also prudent to reintegrate them back to the community (Aziz et al. 2008). Hence, there is a need for the local health system to plan a coordinated transfer of care pathway to ensure continuous medical treatment and support given not only to the patients but also their carers and family.</div>\r\n<div>\r\n	Therefore, information from this study can be used to improve this transitional process and also community stroke management especially during the rehabilitation stages. It is hoped that by identifying the prevalence of the risk factors of stroke and factors associated with its functional recovery, primary and secondary prevention strategies of stroke can be enhanced. This is vital in the effort to restructure the medical care organisation and support network for stroke patients beyond acute in hospital care to the community.</div>\r\n<div>\r\n	ACKNOWLEDGMENT</div>\r\n<div>\r\n	The authors acknowledge Universiti Kebangsaan Malaysia for providing the fundamental research grant and center for data collection.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1048, 1041, '<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 39-year man complained of pain, shortness of breath and bleeding over left anterior chest wall after being stabbed with a small knife. He was instantly rushed to the Emergency Department.</div>\r\n<div>\r\n	The victim was pallor and tachypnoeic with rate of 26 breaths per minute. Initial blood pressure was 80/40 mmHg, pulse rate of 110 beat per minute and saturation under room air was 94%. A 2 cm wound was visualized at the 5th intercostal space medial to the midclavicular line. Reduced chest expansion and dullness percussion was noticed on the left side of the chest. There was no muffled heart sound and jugular venous pressure was not elevated. Abdomen was soft and non tender.</div>\r\n<div>\r\n	Serial Extended Focused Assessment of Sonography in Trauma (EFAST) was performed. E-FAST revealed left side pleural effusion and accumulation of pericardial fluid measuring 1.3 cm anteriorly and 0.8 cm posteriorly. Electrocardiography showed sinus tachycardia with no poor R wave progression. Chest X-ray revealed left hemothorax with haziness over left woundlower zone of the lung. The heart was globular in shape. The mediastinum was normal in size. Blood investigations were normal.</div>\r\n<div>\r\n	He was resuscitated with intravenous crystalloid, colloid and two pints of whole blood. A 32 French chest tube was inserted to the left chest and drained only 250cc of blood. Vitals still showed class three hypovolemic shock, of a BP 80/40 mmHg and pulse rate of 110 beats/ minute. Pulse oximetry was 99-100% on high flow mask. Since echocardiography revealed no pericardial tamponade, pericardiocentesis was not attempted. Repeated FAST was done and did not show any other intraabdominal bleeding. Source of the bleeding could not be determined; hence even in this unstable patient a CT (Computed Tomography) was performed.</div>\r\n<div>\r\n	Left lung contusion with haemothorax and minimal loculated pericardial effusion was evident on CT thorax. The patient was then referred to the cardiothoracic team. No surgical intervention was planned. He was then admitted to surgical high dependency ward for close observation and antibiotic treatment. His vitals stabilized and he was discharged on the fifth day of admission.</div>\r\n<div>\r\n	The patient presented two days later to the Emergency Department with shortness of breath and pleuritic chest pain. He had no fever, cough nor any flulike symptoms. There were no complaints of calf pain or unilateral leg swelling, abdominal pain, vomiting, discharge or redness from the chest tube site. His blood pressure was of 130/80 mmHg, pulse of 80 beat per minute and oxygen saturation was 96% under room air. The air entry was reduced on the left side and was dull on percussion, correlated with the healing lung contusion. Left ventricular thrombus was visualized as a hypoechoic mass via the echocardiogram (Figure 1). The contractility was good and no regional wall motion abnormality was detected. The minimal pericardial fluid was still present. The patient was advised to be admitted be he refused and self-discharged at his own risk.</div>\r\n<div>\r\n	Five days later, he arrived again to the Emergency Department with sporadic loss of vision for the past two days. He described it as a curtain being closed in front of his right eye then subsequently left eye which lasted for 15 minutes. The symptoms were associated with pleuritic chest pain and shortness of breath on exertion. Vital signs were normal. Echocardiogram no longer detected the left ventricular thrombus, which assumed to have fragmented and embolized to the retinal artery. Plain CT brain showed no evidence of infarct. He was treated as amaurosis fugax secondary to thromoemboli and was started on Aspirin 150mg daily. Patients symptoms resolved and he was discharged.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The initial presentation of Stage 3 hypovolemic shock (hypotensive and tachycardia) did correlate with the blood lost due to hemothorax. The echocardiogram did not show any tamponade as the right ventricle was not collapsed in the diastolic phase. Minimal pericardial effusion was detected. In a series done by Tayal et al. (2004) patients with penetrating anterior chest trauma, the Focused Assessment of Sonography in Trauma (FAST) examination was sensitive and specific in the determination of both traumatic pericardial effusion and intraperitoneal fluid indicative of injury, thus effectively guiding emergent surgical decision making (Tayal et al. 2004). Both ultrasound and CT scan explained the amount of blood loss that leads to this stage 3 shock. Only 250 ml of blood was drained from the chest tube, which was not indicative of a thoracotomy. After transfusion he stabilized, and classified as a responder, as there was no ongoing blood loss that need to be halted.</div>\r\n<div>\r\n	The second presentation to the ED with shortness of breath raised a few possibilities, which among them is the cardiac thrombus which resulted in pulmonary embolism. Bedside echocardiography in ED revealed a pedunculated mass in left ventricle where the patient was previously stabbed. This is likely to be a thrombus formation in result to the previous injury. Part of the thrombus must have fragmentized into embolus that lodged in the pulmonary vessels. Unfortunately, further workout of this patient at that time was not possible because he took his own discharge.</div>\r\n<div>\r\n	The patient presented again with symptoms of amarousis fugax, a transient loss of vision caused by lodgement of emboli on the retinal artery. Echocardiography at this time did not show any thrombus which has most likely dislodged. CT brain did not reveal any infarct.</div>\r\n<div>\r\n	Kertes et al. in 1983 reported a case of multiple peripheral emboli after cardiac trauma. It was a case of 25-year-old footballer that sustained non-penetrating (blunt) cardiac trauma after a blow to the chest six months before he had bilateral foot pain and numbness. He was then diagnosed to have left ventricular apical thrombus with multiple systemic emboli to both arterial supply of his feet and right coronary artery. He was treated surgically and discharged well (Kertes 1983).</div>\r\n<div>\r\n	Another case report by Neidlinger et al. in 2004 described a case of cardiac thromboemboli complicating a stab wound to the heart. It was a case of 27-year-old man with a single stab wound at left fifth intercostal space, where left anterior thoracotomy and primary repair of left ventricular apical laceration was done. Post operation day 4 complicated with thromboembolic phenomenon secondary to thrombus of the septal and posterior wall, which responded well with intravenous, subsequently oral anticoagulation and discharged well (Neidlinger et al. 2004).</div>\r\n<div>\r\n	Sharma et al. (2000) found that patients with an embolic secondary to left ventricular thrombus suffered a significantly higher mortality during follow-up when compared to those without an embolic event. In addition, the occurrence of an embolic event had an important bearing on survival, rather than the echocardiographic finding of LV thrombus. This implies that any anticoagulant therapy, if it is to impact on survival, probably should be given preemptively, before the appearance of a thrombus on echocardiography (Sharma et al. 2000). To the best of our knowledge, no cases have been reported in which cardiac trauma specifically caused amaurosis fugax.</div>\r\n', 1),
(1053, 1046, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Pain is a common symptom in medical practice, usually associated with advanced illness or other acute or chronic conditions. It is the symptom that most patients and their families fear most. Child birth is associated with severe pain for most women, (Hiltunen et al. 2004). To few, it can be their greatest pain during their lifetimes (Catterall &amp; Mackie 2006). Labour pain is caused by uterine contractions and cervical dilatation. Pain sensation is conveyed through visceral afferent (sympathetic) nerves entering the spinal cord from T10-L1 through L1 levels. In the later phase of labour, perineal stretching causes painful stimuli transmitted through the pudendal nerve and sacral nerves from S2,3,4 levels. This pain increases maternal stress response and may lead to increase in the release of corticotrophin, cortisol, noradrenaline, B-endorphins, and adrenaline. Adrenaline may have relaxant effects on the uterus and may prolong labour. Catecholamine release may also increase maternal cardiac output, systemic vascular resistance, and oxygen consumption. In few cases with preexisting cardiac or respiratory insufficiencies, this excessive catecholamine release may be life-threatening.</div>\r\n<div>\r\n	Epidural, spinal (intrathecal), and combined techniques are the most effective means of pain relief during labour. These are known as regional techniques because pain relief is limited to a specific region in the body. Epidural analgesia is considered as the gold standard for effective pain management in labour (SOGC 2004). Epidural analgesia during labour and delivery encompass the injection of local anesthetic agents such as lignocaine, bupivacaine or ropivacaine and an opioid analgesic agent such as morphine or fentanyl injected into the lumbar epidural space (Catterall &amp; Mackie 2006). The injected agents gradually diffuse across the dura into the subarachnoid space, where they act primarily on the spinal nerve roots and to some extend on the spinal cord and paravertebral nerves. Oftenly combined with epidural analgesia, spinal analgesia involves the injection of analgesic agents directly into the subarachnoid space, resulting in rapid onset of analgesia (Catterall &amp; Mackie 2006). The drug concentration and volume of local anaesthetics, and opioids used for epidural analgesia are tailored to the stages of labour and the parturient&rsquo;s preference or need for analgesia. Commonly during early labour, 12-14 ml of 0.1% bupivacaine with 2 mcg/ml of fentanyl may suffice. These epidural medications are administered into the epidural space to avoid complications related to inadvertent procedures.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	We here report a case of a 37-year-old primigravida, married for three years, who had a history of uterine fibroid and sub-fertility. She was not on any treatment for her uterine fibroid. For this pregnancy, she had history of premature contraction with administration of intra-muscular dexamethasone during her second trimester. At term, she was admitted to our hospital in latent phase of labour with mild uterine contractions. Vaginal examination revealed her cervical was 2 cm dilated. She was observed and transferred to labour room when her os was 4 cm dilated. Artificial rupture of membrane was performed and she was offered epidural analgesia to reduce her labour pain.</div>\r\n<div>\r\n	Patient agreed for epidural analgesia after the attending obstetrician had explained the benefits and possible adverse effects of epidural analgesia to her and her husband. An anaesthetist further counselled the patient and proceeded with epidural catheter insertion after taking written consent. A test dose of 4 ml ropivacaine 0.2% was administered slowly whilst observing for any adverse reactions. Epidural infusion of ropivacaine 0.2%. and 2 mcg/ml of fentanyl was subsequently started at 7 ml/hr. The patient&rsquo;s vital signs were stable with sedation score of 1/10 and pain score of 1/10. Foetal heart rate was between 122-140 beats per minute.</div>\r\n<div>\r\n	Following three hours of active labour, the patient&rsquo;s cervix was fully dilated. She was encouraged to bear down in order to shorten the second stage of labour. She could still feel the tightening and urge to bear down without any pain. Episiotomy was perfomed on crowning of baby&rsquo;s head without any local anaesthesia. After 25 minutes, a vigorous baby boy, crying at birth, with APGAR score 9 in 1 minute and 10 in 5 minutes was delivered. The baby was born healthy with a weight of 2.93 kg. The epidural analgesia infusion was tapered down until episiotomy repair was completed and removed after one hour. Patient and baby were later discharged to General Ward, eventually.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Epidural analgesia during labour is common. Nurses attending to patients on epidural analgesia must possess adequate knowledge about its indications and contra-indications, and have skills in monitoring its effectiveness and potential complications (Dickinson et al. 2003). The nurses should be apt in assessing the sensory level of the epidural analgesia, usually by using an ice cube in a rubber glove in order to determine the epidural block height. If the patient is unable to detect a cold sensation at/or above the nipple line (at T4 thoracic level), the epidural infusion should be stopped immediately, as the epidural block is too high. They must also prepare, check and update the resuscitation equipment and drugs that may be used during labour.</div>\r\n<div>\r\n	Monitoring of the patient&rsquo;s vital signs is also important to ensure their safety in labour. Should there be any fall of systolic blood pressure below 100 mmHg or if the patient becomes sleepy and difficult to arouse, the epidural infusion should be stopped immediately and the attending doctors informed (Capogna et al. 2007). Occasionally during labour, there may be sudden hypotensive episodes. During such instances, while alerting the doctors, the nurses should turn the patient to her side; increase the intravenous fluid flow rate and supplement oxygen via a face mask. For patients with excessive drowsiness due to opioids, naloxone may have to be considered (Kumar &amp; Paes 2003).</div>\r\n<div>\r\n	During the second stage of labour, constant reassurance and encouragement by the nurses to the patient is essential to achieve successful pain management. In addition, patients&rsquo; epidural acceptance during labour may increase the success of pain management (Pugliese et al. 2013).</div>\r\n<div>\r\n	According to Niesen and Jacob (2013), the initiation of labour analgesia has been reported to have a higher degree of maternal satisfaction while in labour compared to conventional pain management techniques. It is known that instrumental delivery is common with the use of epidural analgesia during labour. The nurses should guide the patient to push using the correct technique during these procedures. At all times, the nurse should closely monitor foetal well being and mother&rsquo;s condition to ensure a safe delivery (Hansen et al. 2002).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	We believe that patient&rsquo;s education and readiness to accept epidural analgesia during labour is very important as it enhances the success of an epidural analgesia. Pain management should be individualised to cater to every parturient. Epidural analgesia is the most effective form of intra-partum analgesia currently available in Malaysia. It is particularly beneficial for parturients with medical problems such as hypertension and cardiac disorders.</div>\r\n', 1),
(1056, 1049, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Numerous homemade remedies have been touted beneficial for acne. In fact, natural remedies may be the first or the preferred option advised by elders or peers for treating acne. This may be due to the perception that natural products are safe for the skin and have less potential to cause adverse effects (Magin et al. 2006). Hence, many maybe unaware that certain natural remedies can cause harm resulting in underreporting by patients themselves or failure of physicians to detect such cases.</div>\r\n<div>\r\n	Contact allergy to cinnamon has been described as early in the 1960s when it was noted to cause hand eczema among bakers (Arthur 1960). Lime on the other hand, causes phytophotodermatitis which is a type of dermatitis due to furocoumarin (psoralens) found in certain plants and vegetables. Contact with lime and subsequent exposure to sunlight is responsible for such a reaction (Robert et al. 2008). We here report two female patients who developed contact dermatitis after topical application of cinnamon and lime in an effort to self-treat their acne.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	PATIENT 1</div>\r\n<div>\r\n	A 22-year-old woman of Malay ethnic origin, presented with redness and burning sensation on her face which started two days before consultation. Initially, lesion appeared on her left cheek then gradually involved other parts of her face. On further questioning, she explained that she had applied a homemade natural remedy for her acne using cinnamon, as advised by her grandmother. Cinnamon bark was first powdered and mixed with water to create a paste and applied over the acne lesions and left overnight. Smiting sensation and erythema first appeared on the third day of application. Clinical examination revealed multiple erythematous patches and over cheeks, chin and philtrum. Multiple closed comedones were noted on the forehead (Figure 1a). Lesions was confined to the face and other parts of the body were not involved. There was no significant personal or family history of allergy or atopy.</div>\r\n<div>\r\n	PATIENT 2</div>\r\n<div>\r\n	A 16-year-old girl of Indian ethic origin presented with asymptomatic dark brown pigmentation on her cheeks. She had applied lime juice on both cheeks as a remedy for acne as advised by her mother. Lime juice was applied and left overnight. Lesion was observed the following day after returning home from school. Clinical examination revealed dark brown to black patches on both cheeks. The lesions had ill-defined margin and scales were absent. Multiple open and closed comedones were noted over the cheeks, temple and forehead (Figure 1b). No other skin lesions were noted elsewhere. Personal or family history of allergy or atopy was absent.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Contact dermatitis is an acute or chronic inflammatory reaction occurring secondary to contact with a certain allergen on the skin. Allergic contact dermatitis is a delayed hypersensitivity reaction (type IV) affecting those with previous sensitization. The delayed reaction may take hours up to days after allergen exposure before reactions manifest. This reaction takes place in two phases. Phase 1 is the induction phase, where the immune system is sensitized to a particular allergen while phase 2, triggers the immune response resulting in the manifestation of overt clinical symptoms (Kimber et al. 2002). The distribution of the lesions serves as a clue to the diagnosis of contact dermatitis where the lesions correspond to the areas of contact between skin and the allergen. Linear or splash lesions may be due to contact with fluids while rounded lesions may be a result of application using fingers.</div>\r\n<div>\r\n	The first patient described in this case report, had developed contact dermatitis to cinnamon. Cinnamon (Cinnamomumzeylanicum) is a condiment obtained from the bark of the cinnamon plant. The volatile essential oil (cinnamaldehyde) causes irritation upon contact with the skin and oral mucosa (Tremblay &amp; Avon 2008). Nutmeg, paprika and cloves are some of the other spices that have been shown to cause contact dermatitis (van den Akker et al. 1990).</div>\r\n<div>\r\n	The second patient depicts a case of phytophotodermatitis. The psoralen in lime causes a chemical reaction which sensitizes skin to the ultraviolet rays from the sun. Besides citrus fruits, other plants which can cause a similar reaction include carrot and celery (Robert et al. 2008). Contact with these plants, vegetables or fruits followed by exposure to the sun, results in dark pigmentation, mild inflammation or vesicles in severe cases. Chelitis, perioral and periorbital dermatitis have also been described secondary to contact with lime (Thomson et al. 2007). The reaction may occur within a few hours and the affected area usually heals with hyperpigmentation. This reaction is believed to be secondary to D-limonene found in the peel oil (Matura et al. 2002). Some fragrances and flavourings using citrus peel oils mayalso cause a similar reaction (Duus Johansen 2002)</div>\r\n<div>\r\n	Diagnosis is made based on history and physical examination. Patch test is the gold standard method to confirm the diagnosis of contact dermatitis however it must be remembered that false negative results are not uncommon (Spiewak 2008). The allergen may be identified with certainty when a similar lesion appears on reintroduction of the allergen.</div>\r\n<div>\r\n	The most important step in managing a case of contact dermatitis is to identify and eliminate the causative allergen. Lesions gradually resolve upon removal of the offending agent and may take between two to four weeks for complete resolution depending on the extent and severity of the insult. Emollients may be used to sooth the skin and short term topical steroids may help speed up recovery. More severe symptoms such as vesicobullous lesions or extensive lesions may require a short course of systemic steroid. Post inflammatory hyper or hypopigmentation is a well known complication. Prevention includes avoidance of using cinnamon or any of its derivatives on the skin and washing the parts in contact. Care should be taken after handling lime or citrus fruits by through washing of the hands before getting outdoors to prevent phytophotodermatitis.</div>\r\n<div>\r\n	Patients should also receive counselling about using homemade remedies. Although cinnamon has anti-inflammatory and anti-bacterial properties which form the basis for acne treatment, it needs to be processed to achieve the appropriate concentration before it can be used for this purpose. This process is complicated and is not easily achievable in our homes (Daud et al. 2013).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	There is a need to create awareness among physicians and patients of traditional or homemade remedies which may be harmful to the skin. This could avoid adding insult to injury in an attempt to self-manage skin conditions. Since acne is a common condition affecting almost everyone at one stage of their life, it is prudent to inquire into self-management practices especially if contact dermatitis is suspected. Upon diagnosis with contact dermatitis, adequate management and advice on future avoidance of the offending agent would help prevent similar episodes. Patient education regarding the availability of a wide variety of established treatment for acne needs to be emphasised through community education and counselling in an effort to reduce the incidence of inappropriate self-management for this common condition.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to thank the patients for their consent for publication of the case and images.</div>\r\n', 1),
(1058, 1051, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Hospital beds closure or access block to ward admission is the single most important cause of Emergency Department overcrowding (EDOC) (Forero &amp; Hillman 2008). This could be compounded further in events of a patient surges e.g. disaster. Despite increased political, administrative, and public awareness, EDOC situations continue to rise in frequency and severity (Bond et al. 2007). A surge response entails even greater responses including implementing affirmative measurement in order to mitigate the issue in tackling the situation.</div>\r\n<div>\r\n	We report a case of EDOC and surge management of this situation. Our Emergency Department of UKM Medical Centre (ED UKMMC) receives high volume of patients (&gt;72,000/ year) (Emergency Department 2011). Cases vary from critical to non-critical, pediatric, geriatrics, medical and trauma. Treatment area is divided into dedicated areas of red (critical), yellow (semi-critical) and green (non-critical) zones. Patients who arrive at the ED ED are triaged according to vital signs and red flag signs/symptoms into these three triage categories.</div>\r\n<div>\r\n	Patients are be assessed and necessary treatment is given.Disposition of the patients are either to home, admitted to the wards, admitted to the observation ward or to the mortuary. The red (critical) zone is a 5 bedded area, including 1 pediatric and 1 cardiac bed, the yellow zone bedshas 13 beds area and the observation ward was 13 bedded areas. When all of these beds occupied, any new patients are lodged at the walking space, either on hospital beds or on foldable beds. The situation required surge intervention when patients that arrived to the ED exceeded the capacity of all available beds. The green zone are 6 cubicle areas which are wall partition convertible to individual room and sees walk in patient. This area receives an average of 150-180 cases per day.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	Wednesday 21st March 2012 was a historical date for ED UKMMC. Starting from 1500 hours that day we stopped taking all non critical cases that came to our doorsteps. Prior to the historical date, we received increasing number of patients started on Monday (274) and Tuesday (243). On Sunday alone, total number of patients admitted to all ward was 37. On Monday, the planned admission was 37 patients, but since the medical wards are full, only six patients are accepted for admission. This brought a backlog of patients to the following day, where an addition of 16 medical patients that needed admission. Since the admission to ward and patient arrival to ED is continuous, at any one time of the day there were over than 30 medical patients temporarily placed throughout the ED.</div>\r\n<div>\r\n	On Wednesday 21st March after our ED ceased to accept any non-emergency cases, the total registered patients dropped 15% to 173. On Thursday the number dropped even further to 84.</div>\r\n<div>\r\n	Admission to wards was constant throughout the days which were 22 on Wednesday and 20 on Thursday. This cleared up the backlogged patients and made ED more manageable.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	We identified and implemented several key principles that are critical in the management of the surge.</div>\r\n<div>\r\n	RECOGNIZING EDOC</div>\r\n<div>\r\n	The definition of EDOC is an increase in the demands placed on an ED, given the normal capacity in which the ED can maintain standard of care (Forero &amp; Hillman 2008). Several criteria have been used to help define overcrowding; these include ambulance diversion, staffing, availability of beds and ED volumes (Lynn &amp; Kellermann 1991; Schull et al. 2002; Kollek 1990; Graff 1999). In this case EDOC was recognized by the lack of space and portable beds for placement of critical and semi-critical patients in the ED, hence translating to inability to provide necessary patient care.</div>\r\n<div>\r\n	INITIATING ACTION</div>\r\n<div>\r\n	Once the surge causing EDOC has been recognized, disaster plan activation was necessary.</div>\r\n<div>\r\n	The ED consultant and Director of hospital were informed. Nearby hospitals were also informed. Strategies such as capacity enlargement plan have been implemented. The ED has been reorganized to facilitate the overcrowding of patients in the yellow and red zones.</div>\r\n<div>\r\n	MAINTAINING PATIENT FLOW</div>\r\n<div>\r\n	There is a need to maintain flow of critical and semi-critical patients throughout the system and avoid any bottlenecks where possible. Intake of green (non-critical) patients were halted and advised to be treated at nearby hospitals. The non-sick, non-surge patients were attended and discharged to community providers. Inbound EMS patients were diverted to nearby hospitals, e.g Hospital Kuala Lumpur or Serdang. Once the green zone was cleared, patients from the yellow zone can be relocated there. Staff and necessary equipments such as defibrillators and emergency trolley are transferred to the green zone. Reallocating resources specifically permits more space and beds for critical and semi-critical patients.</div>\r\n<div>\r\n	SETTING CLINICAL GOALS</div>\r\n<div>\r\n	Notification of EDOC should prompt review of staff work practices in anticipation of increased workloads. The issue is not to work faster or harder than normal, but is to do the most for the most. The standard of care is not changed and priority patients such as life/limb threat, urgent bedside procedures (analgesia, splitting), disposition and patient comfort are not jeopardized.</div>\r\n<div>\r\n	45</div>\r\n<div>\r\n	Emergency Department Overcrowding (EDOC) Med &amp; Health 2013; 8(1): 42-46</div>\r\n<div>\r\n	DEPLOYING A SURGE TEAM FOR ADVANCE TRIAGE OR FAST TRACT</div>\r\n<div>\r\n	Advance triage role is to divert non-emergency patients to a non-ED destination, provide immediate care necessary. It is to prevent passive reception of patient at triage that can swamp the ED, causing overcrowding and lead to impaired clinical care. Other alternatives such as dissuading ED use through media campaigns and diversion of patients to walk-in clinics have been proposed; however, most evidence suggests these are ineffective strategies (Hutchison et al. 2003; Affleck &amp; Innes 2003). The difference between total diversion and advance triage / fast tract is that the latter was 1) safe and did not appear to provide lower quality of care; 2) because they require less resources and cost-effective (Yoon 2003). Several reports conclude that the operation of an ED fast-track system appears to be efficient, operationally cost-effective, safe, and improves patient satisfaction with care (Yoon 2003). In this case several senior doctors were placed in the secondary triage to provide immediate care and necessary referral to the non-emergency patients even prior to their registration at ED. Patients with pre-existing hospital numbers were registered for medicine only and discharged from the secondary triage.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The most common surge and EDOC confronting EDs is the overwhelming demand on a very busy day and aggravated by access block. This overcrowding of patients has shown to be associated with adverse patient outcomes. We believe that the identification of EDOC strategies can lead to better outcome in disaster or overcrowding situations. These measures managed to bring down the number of patients and improve working space. Future plans include creating a temporary inpatient ward where patients admitted to wards can be lodged.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors are grateful to the supporting staff of the Emergency Department Communication Centre for the invaluable data. No financial assistance was received for this study.</div>\r\n', 1),
(1060, 1053, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Retinal detachment (RD) may be of rhegmatogenous, tractional or exudative types. Exudative RD has a distinct features of &lsquo;bullous&rsquo; appearance, compared to the corrugated retinal detachment seen in the rhegmatogenous type. Causes of exudative RD include posterior uveitis, central serous choroidoretinopathy and hypertensive retinopathy (Gass &amp; Little 1995). Smokers are at risk of developing lung carcinoma. Pulmonary tuberculosis, and adenocarcinoma of the lung should be formost in the list of differentials in patients with miliary opacities on X-ray. Exudative RD secondary to malignant uveal melanoma has previously been reported (Kivella et al. 2001)</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 37-year-old policeman, who heavily smoked presented to eye clinic with complaints of sudden and progressive deterioration of the upper half of his right eye visual field. There were no associated floaters or flashes of light, and he denied preceeding ocular trauma.</div>\r\n<div>\r\n	A month previously, he complained of dry cough with intermittent fever of three weeks duration. X-ray at that time revealed an ill-defined opacity at right midzone with military nodules at both lung fields (Figure 1). He also admitted to significant weight loss recently. Subsequently, the case was treated as pneumonia and started on oral augmentin 625 mg bidaily for one week. His symptoms did not improve despite completing the antibiotic regime. Respiratory consults advised anti TB therapy on the basis of positive Mantoux test, although Zhiel-Nielson stain and sputum culture were negative. The condition remained the same and he complained of further three kilogram of weight loss within a month, associated with lethargy. Within the same week, the gentleman presented with the ocular symptoms.</div>\r\n<div>\r\n	On examination, his visual acuity was counting finger on the affected side, with marked relative afferent pupillary defect. Anterior segment examinations revealed bullous inferior retinal detachment apparent without funduscopy, raising suspicion of masquerade syndrome (Figure 2). No retinal break was discerned despite careful examination. The contralateral eye was normal.</div>\r\n<div>\r\n	Another repeat X-ray showed new findings of generalized nodules in both lung fields, with right hilar and paratracheal lymphadenopathy. Computed tomography of CT thorax showed a large mass at the right hilum compressing the upper bronchus, and causing secondary collapse of the right upper lobe, wih multiple nodules throughout both lung parenchyma. There were also bilateral hilar and mediastinal lymphadenopathies with extensive metastases to both lungs, adrenal glands, liver, and brain, and right choroid (Figure 2).</div>\r\n<div>\r\n	Bronchospcopy and biopsy confirmed the diagnosis of poorly differentiated adenocarcinoma of the lung, which was inoperable due to distant metastases. He was planned for combination of radiotherapy and chemotherapy. Unfortunately, patient passed away within two months of his presentation.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This case illustrates well the masquerade syndrome, whence an advance stage of lung cancer presented with retinal detachment, signifying distant metastases. In this case, the recognition of symptoms and sign may not be able to save the right eye vision, but it is certainly is life-saving for the patient.</div>\r\n<div>\r\n	The term &lsquo;masquerade&rsquo; is used to describe uveitides which are not immune mediated in the unusual way, but which have an underlying primary cause (Zamiri et al. 1997). In this case, metastatic tumour spread from primary small cell lung carcinoma to the right globe caused exudative retinal detachment and minimal vitritis in the eye, mimicking inflammation. Ocular secondaries usually originate from renal and lung carcinoma, (Zamiri et al. 1997) as illustrated in this case. Therefore, our threshold of suspicions for malignancies should be high in atypical ocular presentations of retinal detachment without obvious retinal tear.</div>\r\n<div>\r\n	The associations between smoking and cancer have always been postulated, yet the exact patho- mechanism is difficult to delineate. Yang and colleagues suggest that smokers who develop adenocarcinoma of the lung are as susceptible to the tobacco- containing carcinogens as with the small cell and squamous cell carcinoma (Yang et al. 2002). Furthermore, the same study concluded that the association between adenocarcinoma of the lung and tobacco smoking is stronger than was previously thought (Yang et al. 2002) .</div>\r\n<div>\r\n	The thickening on the posterior aspect of the right globe was highly likely due to choroidal metastases (Skarin 1999). A reported a case of metastatic non-small cell carcinoma that initially presented as choroidal tumour, (Skarin 1999) thus raising a suspicion of primary carcinoma elsewhere. The diagnosis of ocular metastases is based primarily on clinical findings supplemented by imaging studies. A study found that choroids was the sixth most common site for metastatic lung carcinoma, following brain, bone, liver adrenals and lung itself (Kreusel et al. 2002). Although choroidal metastases is common in advanced lung cancer, (Kreusel et al. 2002) screening for at- risk individuals may have no great benefit due to short survival of affected individuals.</div>\r\n<div>\r\n	Our patient was given a combination of radiotherapy and chemotherapy, to induce tumour regression. Treated secondary choroidal tumors showed complete regression within five months of treatment in 84% of cases, while serous retinal detachment resolved in 82% percent of treated metastases within 3.8 months after treatment (Tsina et al. 2005). In this study, proton beam irradiation led to complications such as keratitis, cataract, radiation papillopathy and radiation maculopathy in 56 % of cases (Tsina et al. 2005). Although this percentage was quite high, proton beam irradiation allows retention of globe, achieves a high probability of local tumour control and help to avoid pain (Tsina et al. 2005).</div>\r\n<div>\r\n	Reduce visual acuity, although unilateral may have a major impact in one&rsquo;s life, especially in patients who are generally in poor physical condition such as in cancer patients. In a study to look at the safety and efficacy of high dose external beam radiation therapy in patients with choroidal metastases, complete remission of choroidal tumour was achieved in 72% receiving more than 35.5 Gy and 33% of patients who received less than that (Rosset et al. 1998). Visual improvements was also significantly better in the eyes receiving more than 35.5 Gy of radiation dose (Rosset et al. 1998). Although the dosage was considered high, the improvement in vision following regression of choroidal tumour has a major impact in this group of patients who already have a fatal prognosis.</div>\r\n<div>\r\n	Our patient received radiation to the whole brain including the both orbits. Bilateral orbits radiation is recommended to treat subclinical metastases not seen in imaging modalities, and to prevent occurrence of metachoronous metastases (Rosset et al. 1998).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Despite the increasing number of pulmonary tuberculosis cases, lung cancer should be foremost in mind, especially in individuals who have history of heavy cigarette smoking. The public should also be made more aware of the devastating complications and carcinogenic effect of smoking. Effects of smoking on the health of the smokers, the family and family economy, and the cost of treatment of various cancers attributed to smoking must be emphasized.</div>\r\n', 1),
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(1098, 1090, '<p>\r\n	Obesity is a life-long, progressive, life-threatening, costly, genetically-related, multi-factorial disease of excess fat storage with multiple co-morbidities. Bariatric Surgery is currently the most effective therapy available for morbid obesity and can result in improvement or complete resolution of obesity related co-morbidities including diabetes mellitus (DM) (Buchwald et al. 2009). Current indication for bariatric surgery are BMI of more than 35 with co-morbidities, BMI of more than 40 without co-morbidities, having failed conservative method of weight loss with no serious psychiatric disorders. There are several different types of bariatric surgery which includes purely restrictive procedure such as adjustable gastric bands, sleeve gastrectomy and gastric plication, combined restrictive and malabsorptive procedure such as gastric bypass and finally pure malabsorptive procedure such as mini-gastric bypass, bilio-pancreatic diversion and duodenal switch. The advent of laparoscopic technique has resulted in improvement of its safety profile with a 30 day mortality of 0.1-0.3%, similar to a laparoscopic cholecystectomy. Obesity is the primary risk factor for type 2 DM (Chan et al. 1994). DM is a chronic metabolic disorder which if left untreated may lead to serious complications and death. Until the advent of bariatric surgery, the mainstay treatment for DM in the morbidly obese includes diet control, oral hypoglycaemic agents and insulin. One of the main aims of treatment for DM is to prevent the development and progression of micro and macrovascular disease. A 1 percent reduction in HbA1c results in 14%, 37% and 21% reduction in macrovascular, microvascular and deaths related to DM respectively. Remission of DM is a common effect seen after bariatric surgery in the obese diabetics patients (Buchwald et al. 2009). While the initial believe that remission of DM was due to weight loss and reduction in insulin resistance, current evidence show that remission of DM is apparent even before significant weight loss occurs suggesting a more complex neuro-hormonal regulation involvement. It is believed that several enteral hormones are produced following surgeries like sleeve gastrectomy and gastric bypass. The amelioration of DM which occur sooner (within four days of surgery) than associated weight loss was in fact first noted and reported in 1955 following a subtotal gastrectomy with a loop gastro-jejunostomy (Friedman et al. 1955). The foregut (Rubino et al. 2006), hindgut (Patriti et al. 2005), ghrelin hormones and weight loss are thought to be the main mechanism of action of remission of DM following surgery. Furthermore, it was identified that the main gut hormones involved in the entero-insular axis are GLP-1 (Glucagon-like peptide-1) and GIP (Gastric Inhibitory Polypeptide) which are also called &lsquo;incretins&rsquo;, Whilst ghrelin and PYY (peptide YY) seems to play a less prominent role in glucose homeostasis (Papamargaritis et al. 2013). Currently there is compelling evidence from clinical studies such as the STAMPEDE (Schauer et al. 2014), SOS (Swedish Obese Subject Trial) (Sjostrom et al. 2012) and recent systematic review and meta-analysis (Buchwald et al. 2009) that supported the superiority and effectiveness of bariatric surgery in the amelioration of DM when compared to medical therapy alone. In 2011, a position statement made by the International Diabetes Federation (IDF) recommends surgery for diabetic patients with a BMI of more than 35 whom have not achieve treatment targets with medical therapy and in under some circumstances patients with a BMI between 30 to 35 should be eligible for surgery. Hence, is bariatric surgery the cure for diabetes mellitus? It all depends on how we see and perceived. The definition of &lsquo;cure&rsquo; means complete eradication for life. Based on the overwhelming clinical evidence, beyond doubt complete resolution of type 2 DM is achievable, however as shown in the SOS trial where 50% relapse occurred at 10 years follow-up due to multifactorial reasons, whether this is sustainable life-long is questionable. Still, there are many questions to be answered. One thing which is clear is the important role of bariatric surgery in achieving a significant and sustainable weight loss, partial and complete remission in type 2 DM of close to 80% and other metabolic related problems with significant quality of life and cost economics outcome (Buchwald et al. 2009, Klein et al. 2009).</p>\r\n', 1);
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(1105, 1097, '<p>\r\n	INTRODUCTION</p>\r\n<p>\r\n	The prevalence of chronic kidney disease (CKD) and end-stage renal disease (ESRD) worldwide, ranges from 12.1% to 17.5% (Ingsathit et al. 2010; Centers for Disease Control and Prevention (CDC) 2007; Chen et al. 2009; Ong-Ajyooth et al. 2009). Diabetes mellitus (DM) is the leading cause of CKD, accounting for approximately 30% to 40% of CKD and up to 45% of ESRD (National Kidney Foundation 2004). In Malaysia, diabetic nephropathy contributed to more than half of new dialysis patients since 2003, and this number is progressively increasing (National Renal Registry 2011). In view of the increasing burden of chronic kidney disease, factors influencing the disease progress need to be identified to reduce end-stage renal failure.</p>\r\n<p>\r\n	The average time for diabetics with CKD3 to progress to CKD stage 4 is about three years (Meguro et al. 2009). Previous studies have shown that certain risk factors are associated with accelerated progression of CKD including older age, ethnicity, cigarette smoking, duration of diabetes, obesity, systemic hypertension, poor diabetes control and various levels of albuminuria (Meguro et al. 2009; Hsu et al. 2003; Keane et al. 2003; Orth &amp; Hallan 2008).<br />\r\n	A retrospective cohort study conducted by Chia and Ching (2012) in the primary care setting focused on the role of hypertension in development of chronic kidney disease. This study found that older age, diabetes, hyperuricaemia and lower eGFR status was associated with the development of new onset chronic kidney disease. The aim of the present study was to identify the risk factors associated with CKD stage 3b and the effect of maximal dose of angiotensin blockade.</p>\r\n<p>\r\n	MATERIALS AND METHODS</p>\r\n<p>\r\n	SETTING<br />\r\n	This was a pilot unmatched case-control study conducted in a teaching primary care centre in Kuala Lumpur. Prior ethical approval was obtained from the Ethics Committee of UKMMC. A total of 25 cases of patients who had established CKD stage 3b (GFR between 30-45ml/min/1.73m2) and 103 controls of patients with GFR of more than 45ml/min/1.73m2 in the year 2012 were identified from the primary care clinic diabetic registry. Systematic random sampling was used, where every fifth patient in the registry was selected.</p>\r\n<p>\r\n	INCLUSION AND EXCLUSION CRITERIA<br />\r\n	Diabetic patients who were on regular follow-up for at least five years at the primary care clinic were included. Patients with impaired fasting glucose or impaired glucose tolerance, those who developed CKD stage 4 and 5 on or before 2012, pregnant patients at any time during the 5-year of follow-up and patients with other causes of chronic kidney disease such as autoimmune disease, obstructive uropathy, congenital kidney disease as well as drug-induced nephropathy, were excluded from this study.</p>\r\n<p>\r\n	DATA COLLECTION<br />\r\n	Information on socio-demographic data, body mass index (BMI), systolic blood pressure (SBP) and the use of ACEIs or ARBs were collected from patients&rsquo; diabetic records. Dosages of angiotensin-blockade medications were further classified into submaximal and maximal dose. The glycosylated haemoglobin concentration (HbA1c) and serum creatinine levels were traced from the computerized clinical medical information system. Patients&rsquo; case files were reviewed for data the duration of DM.</p>\r\n<p>\r\n	The mean BMI over the five year period was calculated for analysis. The mean SBP and HbA1c of the first reading each year from 2008 to 2012 were used for statistical analysis. Albuminuric status was defined as the latest investigated urine albumin within the five years, prior to recruitment and categorized into normoalbuminuric, microalbuminuric and macroalbuminuric. The first serum creatinine levels for each year from 2008 to 2012 were recorded.</p>\r\n<p>\r\n	CLASSIFICATION OF CKD<br />\r\n	When eGFR declines to less than 60 mL/min/1.73m2, half of the renal function is lost and the prevalence of the complications of CKD, such as hypertension, anaemia, malnutrition, bone disease, neuropathy, and decreased quality of life, begins to rise (Cockcroft &amp; Gault 1976; Swedko et al. 2003; Levey et al. 2003). In this study, CKD stage 3b was selected as the outcome variable because it signifies the definite presence of pathological reduction of GFR (Levey et al. 2003). Studies have shown that there is a steep increase in risk of mortality in patients who have CKD stage 3b compared to with those having CKD stage 3a (Levey et al. 2011).<br />\r\n	The estimated glomerular filtration rate (eGFR) was used to determine renal function. In this study, eGFRwas calculated using Modification of Diet in Renal Disease (MDRD) equation and expressed in units of ml/min/1.73m2 (Levey et al. 2003).</p>\r\n<p>\r\n	STATISTICAL ANALYSIS<br />\r\n	All statistical analysis was performed using the IBM Statistical Package for Social Sciences (SPSS) version 21.0. Normally distributed data was described using mean and standard deviation, whereas skewed data was described using median and interquartile range. Univariate analysis was done using Chi-square, Fisher Exact test and Student t-test for parametric data, and Mann-Whitney U-test was used for non-parametric data. Multiple logistic regression was used to determine the association between the variables and the outcome.</p>\r\n<p>\r\n	RESULTS<br />\r\n	A total of 865 diabetic patients were identified to have continued follow up care in the last five years at PPP-UKMMC. Only twenty five of them had developed CKD stage 3b. Since this number was small, the ratio of controls for each case was increased to 4:1 in order to improve the power of the study.<br />\r\n	Table 1 showed the socio-demographic and clinical characteristics of the patients. They were significantly older (p=0.009) and had diabetes for a longer duration (p=0.002). They were more frequently prescribed with angiotensin blockade medications at the maximum dosage (64.0% p=0.001), had higher systolic blood pressure (155.2 mmHg vs 140.8 mmHg, p&lt;0.001) and lower baseline GFR (64.6 vs 92.4 ml/min/1.73m2, p&lt;0.001) compared to the controls.<br />\r\n	Multiple logistic regression using enter method was used to determine factors associated with the development of CKD stage 3b. The independent variables that were included into the model for multiple logistic regression were age, duration of diabetes, dosage of ACEI/ARB used, BMI, SBP, HbA1c, albuminuric status and baseline GFR. Use of ACEI/ARB could not be entered into the model for multiple logistic regression because all cases were on ACEI/ARB. Due to missing data, only 23 cases and 73 controls were retained for multivariate analysis.<br />\r\n	The logistic regression model was statistically significant (X2(8)= 57.7, p&lt;0.001). The model explained 68.5% (Nagelkerke R2) of the variance in development of CKD stage 3b among diabetics. We were able to correctly classify 89.6% of the cases, with a sensitivity of 72.7% and specificity of 94.6%. Three of the nine variables were found to be significantly associated with the development of CKD stage 3b, namely systolic blood pressure, baseline GFR and use of maximal dosage of angiotensin blockade agents. The results were tabulated (Table 2).<br />\r\n	Every increase of 1 mmHg in systolic blood pressure would increase the risk of developing CKD stage 3b by 8% (Adjusted OR=1.08, 95% CI= 1.02-1.14, p= 0.013). Conversely, every reduction in baseline GFR would increase the risk of developing CKD stage 3b by 11% (adjusted OR 0.90, 95% CI= 0.85-0.95, p&lt;0.001). The use of maximum dosage of angiotensin blockade reduced the risk of developing CKD stage 3b by 7.2 times (adjusted OR=0.14, 95% CI=0.03-0.78, p= 0.025).</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	Systolic blood pressure is an established risk factor for the development of chronic kidney disease (Haroun et al. 2003; Bakris et al. 2003; Ingsathit et al. 2010; Hooi et al. 2013). The finding of this study was consistent with earlier studies, supporting the importance of blood pressure control in preventing renal complications among diabetics. Unfortunately, only a quarter of patients with diabetes in the primary care setting manage to achieve targets for blood pressure control (Cheong et al. 2013). This places Malaysian diabetics at higher risk of developing chronic kidney disease.</p>\r\n<p>\r\n	Hypertension and chronic kidney disease form a vicious cycle. Hypertension is a proven predictor of development of renal impairment, whereas presence of renal impairment increases blood pressure and affects blood pressure control (Bakris et al. 2003; Haroun et al. 2003; Sarafidis et al. 2008). Despite the variability of GFR decline, progression to end stage renal disease is increased whenever the baseline GFR is lower (Li et al. 2012). The findings of the present study were similar to an earlier cohort study which looked into trajectories of GFR among patients with CKD (Li et al. 2012).<br />\r\n	Many landmark trials demonstrated the benefits of angiotensin blockade medications in delaying the progression of diabetic nephropathy (Heart Outcomes Prevention Evaluation Study Investigators 2000; Ravid et al. 1998; The Diabetes Control and Complications (DCCT) Research Group 1995). Our findings confirmed that by optimising the dose of angiotensin blockade among hypertensive diabetics, the risk of developing severe chronic kidney disease was reduced by seven times. Although the use of angiotensin blockade agents among diabetics with hypertension have increased in the recent years, there is no published data regarding the dosage of such agents used in the local population (Chan 2005; Chew et al. 2010). There is a need to increase awareness to family physicians that the renoprotective effects of ACE inhibitors and angiotensin receptor blockers are best when maximum recommended dosage is used. However, this practice needs to be done with monitoring to avoid problems such as angiotensin-blockade related renal failure and hyperkalaemia.</p>\r\n<p>\r\n	A cross-sectional study in Malaysia showed that prescription of ACE inhibitors and ARBs were still very low among diabetic patients in 2003 (Chan 2005). Another study showed that 68.1% of diabetics were on ACE inhibitors, whereas 7.6% of diabetics were on ARBs (Cheong et al. 2013). This could be attributed to higher adoption of local clinical practice guideline by physicians. The improvement in prescribing patterns for such agents is thought to reduce the incidence of diabetic nephropathy and new-onset chronic kidney disease.</p>\r\n<p>\r\n	Currently, there is no published data regarding the proportion of diabetic patients who are being prescribed the maximum dose of ACEI or ARBs. Clinical audits should be done in order to study the prescribing patterns of such agents in order to maximize its benefits on reducing the risk of developing severe chronic kidney disease.</p>\r\n<p>\r\n	This study was carried out in a primary care setting, where development of chronic kidney disease is usually first diagnosed. The clinical profiles of patients in a primary care clinic are different from those found in secondary or tertiary care, which caters to patients with more complicated problems. CKD stage 3b is usually detected in primary care clinics. Intervention at this stage could delay the progression to more severe stages of CKD.</p>\r\n<p>\r\n	Limitations of this study include the small sample size due to lack of eligible cases. Therefore, this study may be underpowered to detect significant associations for other factors such as age, duration of diabetes, HbA1c and BMI. However, clinicians should continue to address modifiable risk factors such as glycemic control and body weight among diabetic patients who are at risk of developing CKD. Secondly, the baseline characteristics of cases and controls were different in many aspects. Therefore, multiple logistic regression was used in order to control for the effect of confounding factors. All cases were on ACE/ARB in this study. The researchers were unable to analyse whether use or non-use of ACEI/ARB affected the development of CKD stage 3b. Being a retrospective study, there were some problems of missing data. Hence, a definite causal relationship between the associated factors and the development of CKD stage 3b could not be demostrated. Patient adherence to ACEI/ARB, interaction with other medications and possible concomitant use of nephrotoxic drugs were other possible confounding factors which could not be addressed in this study.</p>\r\n<p>\r\n	It is recommended that predictors of CKD in the primary care setting are studied further by conducting a multi-centered, cohort study in order to further establish the relationship of maximal dose of ACEI/ARB with delaying onset of CKD stage 3b among diabetics.</p>\r\n<p>\r\n	CONCLUSION<br />\r\n	Systolic blood pressure and lower baseline GFR are significantly associated with the development of CKD stage 3b in the primary care setting. The use of maximum dosage of angiotensin blockade agents was found to have protective effect towards the development of CKD stage 3b. Primary care doctors should aim for better control of blood pressure among and to optimise the dose of angiotensin blockade agents among diabetic patients. However, use of maximum dose of such agents should be done with appropriate monitoring to prevent drug-related adverse events.</p>\r\n<p>\r\n	ACKNOWLEDGEMENT<br />\r\n	The authors would like to acknowledge UKMMC for funding the present study (UKMMC Fundamental Research Fund (Project code FF-064-2013) as well as clinic staff of UKMMC Primary Care Centre for their assistance in data collection.<br />\r\n	&nbsp;</p>\r\n', 1),
(1109, 1101, '<p>\r\n	INTRODUCTION<br />\r\n	Clostridium difficile is recognized as a significant nosocomial pathogen responsible for antibiotic-associated diarrhoea in most hospitals worldwide (Bartlett 1996; Hall &amp; O&rsquo;Toole 1935; McFarland 1998). It is an opportunistic, gram-positive, rod-shaped, spore-forming anaerobic bacterium known to be one of the minor colonic floras. The diagnosis of Clostridium difficile infection (CDI) is based on the presence of diarrhoea and stool test positive for toxigenic C. difficile or its toxin or colonoscopy or hisptopathologic findings demonstrating pseudomembranous colitis (Cohen et al. 2010).</p>\r\n<p>\r\n	The prevalence of CDI varies with hospital populations. In the United States, it affects over three million diarrhoeal and colitis patients, annually (Schroeder 2005). In Asia Pacific region, CDI prevalence had increased over the years. In Singapore for instance, the prevalence increased from 1.49 cases per 10,000 patient-days in year 2001 to 6.64 cases per 10,000 patient-days in year 2006 (Lim et al. 2008). In Malaysia, three out of seven CDI cases resulted in death from pseudomembranous colitis (Parasakthi et al.1988). Recently, it has been observed that incidence of CDI cases in northern eastern coast of Malaysia is 13.7% (Hassan et al. 2012).<br />\r\n	Although CDI had been extensively investigated in most western and Asia Pacific region, there has been no established data on it at Universiti Kebangsaan Malaysia Medical Centre (UKMMC). The aim of the present study was to determine the incidence, prevalence and epidemiological and clinical characteristics of CDI among hospitalized patients at UKMMC.</p>\r\n<p>\r\n	MATERIALS AND METHODS</p>\r\n<p>\r\n	STUDY DESIGN<br />\r\n	The cross-sectional study was carried out in Bacteriology Laboratory, Department of Diagnostic Laboratory Services from November 2011 until October 2012. All unformed stool specimens from patients suspected with CDI as sent by clinicians were included. Duplicate specimens from the same patient within a 10-day period were excluded from this study (Peterson &amp; Robicsek 2009). The diagnosis of CDI was confirmed by presence of C. difficile toxins in stool.</p>\r\n<p>\r\n	CLOSTRIDIUM DIFFICILE TOXIN DETECTION<br />\r\n	Clostridium difficile toxins A and/or B were detected by a commercial kit (Wampole&trade; Tox A/B QuikChek, Techlab, USA), having 90.2% sensitivity and 99.7% specificity (manufacturer&rsquo;s insert). This rapid membrane enzyme immunoassay uses specific antibodies to detect toxins A and/or B of C. difficile in stool. The procedure steps were followed as directed. Presence of blue line in control and test reaction window within a 10-minute reading time was interpreted as positive result; indicating the presence of C. difficile A and/or B toxin.</p>\r\n<p>\r\n	DEMOGRAPHIC AND CLINICAL DATA</p>\r\n<p>\r\n	Data of C. difficile toxin-positive cases that were collected from patients&rsquo; medical records included length of hospitalization, onset of diarrhoea, antibiotics used two months prior to diarrhoeal onset, nasogastric tube usage, immunosuppressive therapy, recent surgical intervention, underlying disease and others.<br />\r\n	Monthly data on inpatient-days were obtained from Medical Informatics Department to calculate the incidence of CDI. The study received approval from the Research and Ethics Committee of Universiti Kebangsaan Malaysia (ERGS/1/2011/SKK/UKM/03/29).</p>\r\n<p>\r\n	STATISTICAL ANALYSIS<br />\r\n	Statistical analyses were performed using the Statistical Package for the Social Sciences (SPSS) software version 15.0 (SPSS, Inc, USA). Categorical variables and continuous variables were described as number (percentage) and median (IQR or range), respectively.</p>\r\n<p>\r\n	RESULTS<br />\r\n	A total of 147 stool specimens were collected throughout the one-year study period. Nine of the 147 specimens were C. difficile toxin-positive and categorized as CDI (Figure 1). The overall prevalence of CDI was 6.1%. Monthly incidence of CDI was presented (Figure 2). Cases of CDI occurred sporadically during December-January and May-June. However, there were no CDI in the remaining months. The overall incidence throughout the study period was 5.2 cases per 10 000 patient-days. The average monthly incidence rate was 0.4 per 10 000 patient-days.</p>\r\n<p>\r\n	Demographic and clinical characteristics of nine CDI cases were summarized (Table 1). It was revealed from the table that male to female ratio is 7:2, and affects both Chinese and Malay. The median patient age is 60 years (IQR: 57-67.5 years, range: 25-74). Majority of cases are from medical wards.<br />\r\n	Two (22.2%) of the diarrhoeal cases occurred prior to the hospitalisation and confirmed CDI within 48 hours of admission. The remaining seven cases acquired CDI during hospitalisation. Median duration between admissions to CDI onset was six days (IQR: 6-12 days, range: 5-29 days). Median time to CDI detection was nine days (IQR 6-13 days, range: 6-32 days).<br />\r\n	One (11.1%) of the nine cases did not have antibiotics therapy eight weeks prior to diarrhoea onset. In the remaining eight cases, a combined total of 22 courses of antibiotics (median: 2, range: 1-5 courses) and 106 days of antibiotic therapy (median: 13.5, range: 5-21 days) were implicated. Penicillin-beta lactamase inhibitors group (Table 2) was the most common antibiotics used; accounting for 49 out of 106 days. Cephalosporins and carbapenems accounted for seven and six out of 106 days, respectively.</p>\r\n<p>\r\n	Records on prior hospitalisation were available in seven CDI cases. Four (57.1%) of these cases had hospitalization 12 weeks prior to the current admission. There were no recurrent CDI found in this study. The median length of hospitalisation for CDI was 13 days (IQR: 8-18 days, range: 7-39 days). Most cases had two or more underlying diseases. Five (55.6%) of the cases were on acid-suppressing medication. Throughout this period, majority of CDI was mild to moderate. None of the cases was treated with oral metronidazole or oral vancomycin. Hospital mortality rate among CDI cases was 22.2%.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	The overall prevalence of CDI at UKMMC was 6.1% and the overall incidence was 5.2 cases per 10 000 patient-days. This data is comparable with the data from southern Taiwan and Singapore (Chung et al. 2010; Lim et al. 2008). Our prevalence was substantially lower than that of Canadian hospitals (Simor et al. 2013) and northeastern part of Peninsular Malaysia (Hassan et al. 2012). The monthly incidence rate indicates that CDI is not endemic in our institution.<br />\r\n	The difference in prevalence and incidence can be explained by (1) difference in hospital categories (2) diversity in study population (3) variable study designs and sampling methods and (4) prior medications and therapeutic intervention (Chung et al. 2010). In our institution, low prevalence of CDI could be due to lack of awareness to CDI (Jamal et al. 2010). Lack of suspicion affects the yield of sampling and reduces the chances to diagnose CDI. In addition, the use of less sensitive technique instead of tissue culture cytotoxin assay and toxigenic C. difficile culture (Alcal&aacute; et al. 2008) may also explain for this finding. Hence, we started with another study involving several sensitive techniques to validate the current findings.</p>\r\n<p>\r\n	Major risk factors for CDI are hospitalisation, older age and history of antibiotic usage (Bartlett 2008). Underlying disease has been reported as another major risk factor for nosocomial CDI (Kyne et al. 2002). In our study, we observed that majority of CDI cases had diarrhoea onset during hospitalisation and six (66.7%) of these cases were patients older than 65 years old. In addition, eight (88.9%) patients had antibiotics therapy within eight weeks before diarrhoea onset and all nine cases had at least one underlying disease. Unlike in other studies, tube feeding and oesophagogastroduodenoscopy (OGDS) were not common therapeutic interventions among CDI cases in our institution (Bliss et al. 1998; Thibault et al. 1991). More than half of CDI cases had received acid-suppressing medications within 8 weeks before the diarrhoeal onset. This finding is similar to that reported by Aslam &amp; Musher (2006). Our study did not demonstrate significant association between CDI and the established risk factors (data not shown) because of small number of cases studied over a short period. To identify the risk factors in our institution, a case-control study involving larger samples would be appropriate.</p>\r\n<p>\r\n	For hospital-acquired CDI the median duration of diarrhoea onset was six days and median time to detection was nine days. Our study had shorter median time for detection as majority of the cases had earlier diarrhoea-onset between five to six days. Previous study by Forster et al. (2012) showed a longer time for detection of hospital-acquired CDI as the study focused on cases occurring after day-7 hospitalisation.<br />\r\n	It was observed that penicillin-beta-lactamase inhibitors were the most common antibiotics received prior to CDI onset. This finding is similar to the previous study by Lee et al. (2012). Unlike in other studies, cephalosporins were not the common antecedent antibiotics (Kim et al. 2014; Lai et al. 2013). We also found that most of the cases were not on &lsquo;low risk&rsquo; antibiotics prior to CDI onset. To reduce CDI incidence, Talpaert et al. (2011) recommended the usage of &lsquo;low risk&rsquo; antibiotics for empirical treatment of common infections among hospitalized patients.<br />\r\n	The median length of hospitalization for CDI cases was 13 days. Our finding is consistent with other studies. Few studies found that CDI is more likely to occur among patients who are hospitalized for seven days or more (Dumyati et al. 2012; Lee et al. 2012; Manian et al. 2007). We found that majority of the cases were mild to moderate and did not require oral metronidazole or oral vancomycin. Two cases that succumbed to death were related to their underlying primary diseases. However, in one case, the patient died one day after CDI was diagnosed. The death could be attributed to the complications associated with CDI.</p>\r\n<p>\r\n	A continuous CDI surveillance is useful to illustrate future incidence trend, disease recurrence and changes in our local clinico-epidemiology pattern. Based on this observational study, physicians should keep CDI in consideration in all cases of diarrhoea that had antibiotic therapy and/or acid suppressing medications in the preceding eight weeks.</p>\r\n<p>\r\n	CONCLUSION<br />\r\n	In conclusion, the incidence and prevalence of CDI in our institution were relatively low. The cases occurred sporadically and were mild to moderate in severity. Our preliminary data has important clinical significance that conveys strong message: community and hospital-acquired CDI do exist at our institution. A continuous surveillance and regular antibiotic audit may help curb CDI from spreading further.</p>\r\n<p>\r\n	ACKNOWLEDGEMENTS<br />\r\n	The authors wish to thank Shaliawani Mohd Puzi, Nor Afnizan Mohd Yusof and the staff in Bacteriology Laboratory, Department of Diagnostic Laboratory Services for their technical support throughout the study. This study was a part of CDI project funded by Exploratory Research Grant Scheme - ERGS/1/2011/SKK/UKM/03/29.</p>\r\n', 1),
(1116, 1108, '<p>\r\n	INTRODUCTION<br />\r\n	As nursing practice continues to increase in complexity and nurses assume greater responsibility and accountability in the delivery of care, nursing students must be well-equipped with knowledge and skill in rendering nursing care for patients in the clinical setting (Gordon et al. 2013). To be able to perform competently in their roles, nurses must be prompt and able to identify abnormalities in the signs and symptoms of the patients. It can be argued that the primary aspect to achieving this outcome is the role and responsibility of nurse educators and other team players, such as clinical instructors, registered nurses and faculty of the medical setting, who are responsible for creating an adequate platform to foster student learning (Caldwell et al. 2010).<br />\r\n	The nursing students&rsquo; role and responsibility in the clinical setting is to plan nursing care that addresses patients&rsquo; needs in a holistic, individualized manner through the use of the nursing process. The nursing process format requires that nursing students systematically collect and scientifically analyze the patient&rsquo;s unique data as a basis to identify nursing problems in order to make diagnoses, plan and implement nursing interventions, and evaluate the response to those nursing interventions (Koehler 2004). The nursing care plan can be revised according to assessment of new data to meet the patients&rsquo; needs. Nursing students develop care plans and make clinical decisions based on their theoretical and scientific body of knowledge to set priorities and implement care. Bucknall (2003) highlighted the clinical landscape of critical care on nurses&rsquo; decision making. It was reported that the three main environmental influences that affected decision making among nurses were patient situation, resource availability and interpersonal relationships. The research concluded that decision making is a manifestation of the hospital setting and patients&rsquo; needs. However, nurses&rsquo; prerequisite knowledge and skill play an important role in nurses&rsquo; decision making to improve nursing outcomes. Certainly, experiential knowledge and skill will contribute to positive clinical decision making by nurses (Horton et al. 2012).<br />\r\n	Keeping the above facts in mind, the present study involved the use of a modified version of CDMNS questionnaire. The main purpose of this study was to examine the perception of decision making ability of nursing students in a tertiary hospital in Malaysia.</p>\r\n<p>\r\n	MATERIALS AND METHODS<br />\r\n	This was a cross-sectional study conducted in Universiti Kebangsaan Malaysia Medical Centre (UKMMC) from June 2007 till December 2007. Nursing students in their final year of the Bachelor of Nursing degree programme in UKMMC were recruited after informed consent was obtained. Those who refused to give their consent were excluded.<br />\r\n	Prior ethical approval was obtained from the Universiti Kebangsaan Malaysia Research Ethics Committee. Prior permission was obtained for this study from the director of the Universiti Kebangsaan Malaysia Medical Center (UKMMC). This study was supported by the Universiti Kebangsaan Malaysia (UKM) grant (FF-243-2007).</p>\r\n<p>\r\n	INSTRUMENT<br />\r\n	The CDMNS targets decision making processes viewed as essential cognitive skills for professional nurses (Jenkins 1985). Students rate their behaviors related to decision making while providing patient care in the clinical setting. A total of 54 nursing students from department of nursing UKMMC were recruited. A modified version of CDMNS was used to measure the nurses&rsquo; clinical decision making ability. A 40-item questionnaire was used to measure the nursing students&rsquo; clinical decision making. The scores were rated on a 5-point Likert scale (1=never, 2=seldom, 3=occasionally, 4=frequently and 5=always). The instrument provides an overall score for clinical decision making and four subscale scores, which include: search for alternatives and options scale, canvassing of objectives and values scale, evaluation and re-evaluation of consequences scale, and search for information and unbiased assimilation of new information scale.</p>\r\n<p>\r\n	Summation of score was classified as follows: 40 was considered low, and 200 considered as high. The higher the score indicated the higher ability of the nursing students&rsquo; clinical decision making. International consistency was found using Cronbach&rsquo;s alpha (r=0.70), which was adequate. The Cronbach&rsquo;s alpha for this instrument ranged from .71 to .81, with overall coefficient of .74. Socio-demographic data included age, education level, nursing as first choice, and satisfaction in nursing.</p>\r\n<p>\r\n	<br />\r\n	STATISTICAL ANALYSIS<br />\r\n	Statistical Package for Social Science (SPSS) version 21 for Windows was used for statistical analyses. Descriptive analyses included frequency of the respondents&rsquo; socio-demographic, mean and standard deviation on the CDMNS score. T-test and Analysis of Variance (ANOVA) tests were used to compare the relationship between socio-demographic profile and CDMNS score of the participants.</p>\r\n<p>\r\n	<br />\r\n	RESULTS<br />\r\n	Respondents&rsquo; socio-demographic data included age, education level, nursing as first choice, and satisfaction in nursing (Table 1). The participants&rsquo; age groups that ranked the highest was 19 to 23 years with 23 (43%), 24 to 29 years with 2 (4%), and more than 30 years with 29 participants (53%). Education level of the participants showed that 30 participants (56%) had studied until high school, 9 participants (17%) until pre-university level, and 15 participants (27%) until foundation program. Nineteen participants (35%) stated nursing as their first choice, whereas 35 (65%) participants did not. 37 participants (69%) were satisfied in their nursing practice, while 17 (31%) felt dissatisfied.<br />\r\n	The findings showed good CDMNS score with mean and standard deviation of (124.24&plusmn;12.713). The results of the four CDMNS sub-scales were: searching for alternative (33.24&plusmn;4.821), canvassing (28.74&plusmn;3.514), evaluation and re-evaluation (31.43&plusmn;3.922), searching for information (30.83&plusmn;4.765) (Table 2).<br />\r\n	There were significant differences between searching for alternatives, evaluation and re-evaluation, and nursing as their first choice (p=&lt;0.05). In terms of nursing as first choice and searching for alternatives for clinical decision making, showed that those who did not choose nursing as their first choice scored higher (34.46&plusmn;4.907), while those who chose nursing as their first choice scored lower (31.00&plusmn;3.844), with (t=-2.856; p=0.006). However, there were no significant differences between canvassing and searching for information, and nursing as their first choice (Table 3).<br />\r\n	There were also significant differences between searching for alternatives and those satisfied with nursing competency with mean and standard deviation of (34.78&plusmn;4.315), participants unsatisfied with nursing competency (29.88&plusmn;4.196) with (t=3.951;p=&lt;0.05) (Table 4).<br />\r\n	There was significant difference between education level and searching for alternatives and information (p=&lt;0.05). Participants with high school qualifications with mean and standard deviation of (35.77&plusmn;3.971), pre-university (29.67&plusmn;4.975) and foundation (30.33&plusmn;3.200) with (F=13.830;p=&lt;0.05) (Table 5).</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	The results of the study showed that the nursing students possessed adequate clinical decision making ability in the clinical setting. The nursing curriculum that has been well-structured with comprehensive content and effective teaching methodology allow the nursing students to integrate learning into their clinical practice. In addition, the tertiary hospital is well-equipped with the latest technologies and experienced faculty staff to guide and supervise nursing students in the clinical practice setting. del Bueno (2005) reported that new nursing graduates lack the ability to make clinical decisions upon joining the nursing workforce. Conversely, Wiles et al. (2013) argued that newly graduated nurses more often than not are thrust into roles they are not prepared to handle. In the local scenarios from the study have showed that the nursing students of this tertiary hospital possessed committed lecturers and adequate hospital facilities to fulfill the learning needs of the students.<br />\r\n	In this study, the results revealed that searching for alternative solutions is one of the major attributes when making clinical decisions. In the hospital setting, decision making has to be prompt and appropriate action must be taken in order to prevent further deterioration of the patient&rsquo;s condition. This shows that nursing students are capable of seeking solutions and help to overcome difficulties in clinical decision making. Interestingly, in this study, it can be seen that there were significant differences in CDMNS scores for alternatives in terms of education level. Those with SPM education were noted to have better scores than STPM or matriculation. This suggests that the higher the education level, the less likely they would search for alternatives. This needs to be investigated further. However, the older they are, the better the CDMNS score, suggesting maturity in thinking as they search for alternatives. This needs to be validated further.<br />\r\n	Congruently, a study conducted by Smith et al. (2013) reported that additional education interventions were necessary to promote confidence in triage decision making among final semester nursing students. Aitken et al. (2009) stated that decision making in the clinical setting involves integration of a wide range of attributes in the assessment aspect of patient care. Their research concluded that clinical decision making should integrate a closed clinical supervision and guidelines to achieve positive patient outcomes.<br />\r\n	Nursing students undergoing clinical practice were closely supervised throughout their posting by well trained preceptors. The preceptors&rsquo; knowledge and skillful bedside teaching and clinical skills could have assisted further in influencing their confidence and competency among nursing students in this tertiary hospital, which may contribute to the development of their clinical decision making ability. The nurse-patient ratio in the medical and surgical unit is 1:5, therefore nursing students are able to be more focused on their patients&rsquo; health condition and make sound clinical decisions with the supervision of the registered nurses who act as preceptors in the clinical setting. The preceptors in the wards have undergone preceptorship training programs and are well-prepared to supervise and guide the students. Should the nursing students make any mistakes, it can be identified and remedied immediately without causing harm to the patients.According to Bj&ouml;rkstr&ouml;m et al. (2008) it was reported that nursing students considered themselves competent in terms of nursing competence, but their self-judgment reduced in relation to the new demands as a professional nurse.<br />\r\n	Nursing students have to acquire the necessary knowledge and skill related to clinical decision making ability in the clinical setting, and be prepared for the role and responsibilities of delivery of care to patients. The nursing curriculum have great emphasis on the quality of care and competent skills in the delivery of care to equilibrium with the soft skills to meet the needs of patients and family whilst hospitalization. Previous study by Standing (2007) reported that acquisition of clinical decision making skills and preparedness regarding responsibilities as nurses require nursing students to &lsquo;think on their feet&rsquo; without the &lsquo;comfort blanket&rsquo; of student status. The nursing students early exposure to clinical setting could have contributed to their development and improvement in clinical decision making in the real world situation.<br />\r\n	Limitations of this study was that only the final year nursing students in one tertiary education centre were recruited. A larger sample size involving other institutions would be able to validate the findings of this study.</p>\r\n<p>\r\n	<br />\r\n	CONCLUSION<br />\r\n	Clinical decision making ability in nursing is one of the most important skills nursing students must learn and employ in their nursing practice in order to ensure patient safety and optimal delivery of care. In conclusion, nursing students possess adequate clinical decision making ability during their clinical posting. These findings may further assist nurse educators to better plan clinical experiences and more effectively utilize various training to optimize clinical learning in a tertiary hospital.</p>\r\n<p>\r\n	ACKNOWLEDGEMENT<br />\r\n	The authors would like to acknowledge the help from registered nurse, nurse manager and head nurse of UKMMC for their cooperation in responding to the survey. Heartfelt thanks to Dato&rsquo; Prof. Dr. Raymond Azman Ali, Dean, Faculty of the Medicine and Prof. Dr. Srijit Das for their continuous guidance and support.</p>\r\n', 1),
(1120, 1112, '<p>\r\n	INTRODUCTION<br />\r\n	Access to an intravenous (IV) route is very crucial in emergency patients under resuscitation. Immediate actions for vascular access are important to administrate fluids and drugs especially for patients in critical condition with collapsed peripheral veins. Studies show that the failure rates of IV access in emergency situations are described between 10-40% (Lapostolle et al. 2007; Lewis 1986; Minville et al. 2006). Furthermore, accessibility to peripheral IV took an average time up to 2.5-13 minute and more worse even up to 30 minutes in case of vascular collapsed (Costantino &amp; Fojtik 2003; Costantino et al. 2005). Thus, it will delay the treatment of patients (Turner et al. 2000).<br />\r\n	Drugs and fluids can be administered in many others ways such as sublingual, endotracheal, subcutaneous and intramuscular. However, these options are not very effective for patient with hemodynamically inconsistent and only small amounts of suitable drugs are possible to be given. Furthermore, for unstable patients who required IV access but it is impossible at emergency department the other alternative is Central Venous Catheterization (CVC) (Turner et al. 2000; American Heart Association 2005a). Nevertheless, to access a CVC an expert and patient&rsquo;s safety need to be considered. Therefore, an alternative means to access vascular can be done by intraosseous cannulation (AHA 2005b). Its importance in adults is less propagated, especially for in-hospital use. This procedure is not required highly qualified personnel even it is can be done by are paramedics in both; pre and in-hospital setting (Biarent et al. 2005). However, the extent of knowledge regarding intraosseous among emergency paramedics is unknown. Therefore, the aim of this study was to determine the level of knowledge on intraosseous cannulation among emergency paramedics.</p>\r\n<p>\r\n	<br />\r\n	MATERIALS AND METHODS<br />\r\n	A total of 15 emergency paramedics working at one of government hospital were recruited for this study. This was a cross-sectional study conducted using 10 structured questions (one best answer) to test the level of knowledge on intraosseous cannulation among emergency paramedics. They were required to give their consent and answer a short 6 minutes test on intraosseous cannulation knowledge. The questions were based on intraosseous cannulation knowledge which covers guideline, algorithm, indications, contraindication, location for IO puncturing, and steps of procedure. The level of knowledge on intraosseous cannulation will be determined by their score. Score of 75% and above were considered as an adequate knowledge. The questions were prepared in English. A pilot study (pre-test) was conducted on five paramedic students one week prior to the actual research work. Comments and feedback on the questions was considered to obtain a better understanding of the questions. The questions were tested and its reliability was &alpha;&alpha;=0.06. A descriptive analysis was conducted to determine the level of knowledge score.</p>\r\n<p>\r\n	RESULTS<br />\r\n	DEMOGRAPHIC CHARACTERISTICS</p>\r\n<p>\r\n	Total of 15 emergency paramedics participated in this study. Two out of 15 were females, and the rest 13 were males. The age was between 22 to 45 years. The working experiences were from two years until 13 years. Majority of the participants were Malays (93.3%) and Indians (6.6%) in this study.</p>\r\n<p>\r\n	<br />\r\n	KNOWLEDGE ON INTRAOSSEOUS CANNULATION<br />\r\n	Only one one out of 15 participants had 75% knowledge score on intraosseous cannulation followed by 4 out of 15 had 60% score. Majority of the participants scored 40% to 50%. However, there was no participant who scored less than 40% knowledge score on intraosseous cannulation. The results showed only that only one (6.7%) out 15 participants had an adequate knowledge on intraosseous cannulation.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	Paramedics are the health care personnel who involved in emergency care at pre-hospital and in-hospital settings. They should be well equipped with all the necessary emergency knowledge and skills in order to perform their job better. Perhaps, rarely perform skills like intraosseous cannulation also need to be emphasised. In Malaysia, tthis procedure is not frequently performed for patient with difficulty of intravenous access. This is due to less of emphasise on the practicality of this procedure. Furthermore, lack of knowledge as well as the confidence of performing this procedure also contribute to it.<br />\r\n	This study showed that there was only 6.7% from the total of 15 participants had an adequate knowledge on intraosseous cannulation. This was probably due to his/her working experiences in emergency department. By referring to the working experience, this participant was working for more than two years and it could be the contributing factor for him/her to gain some knowledge on this procedure. The rest showed clearly that inadequate knowledge on intraosseous cannulation and matter to increase knowledge on this skills need to be focussed. It is possible to improve the knowledge and skills on intraosseous cannulation among paramedics and it is positively shown with a study by Pfister (2007) that paramedics had performed better in term of success rate at the first attempt than physicians. This suggests that well-trained health care providers could attain an IO cannulation better.<br />\r\n	CONCLUSION<br />\r\n	As a conclusion, the knowledge on intraosseous cannulation among emergency paramedics needs to be improved as evidenced in this study. The lack of knowledge among emergency paramedics may increases the effect on the practices of intraosseous cannulation, which influences the mortality and morbidity of emergency patients. There is a need for education on intraosseous cannulation among paramedics who are working in emergency department.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1122, 1114, '<p>\r\n	INTRODUCTION<br />\r\n	Tuberculosis still poses a significant health problem in Malaysia. Based on WHO tuberculosis report 2013, the incidence rate of tuberculosis in Malaysia is 80/100,000 population and the mortality rate is 5.4/100,000 population (Global tuberculosis report 2013). Genitourinary system is the second most common site of TB following pulmonary system. Genitourinary tuberculosis (TB) contributes 10-41% of the extrapulmonary tuberculosis (Eastwood et al. 2001; Gupta et al. 2008). However, TB affecting the prostate is less common than renal, vesiculo-seminal and epididymal TB, as a result it may post some difficulties in its diagnosis and management. Many cases were found incidentally following transurethral resection of prostate (Gow 1998). Here, we report a case of TB of the prostate presenting as benign prostatic hyperplasia.</p>\r\n<p>\r\n	<br />\r\n	CASE REPORT<br />\r\n	A 70-year-old man was admitted for acute urinary retention. He also gave history of haematuria for the past three days. There was no history of fever, cough or weight loss. He was diagnosed to have lower urinary tract symptoms (LUTS) secondary to benign prostate enlargement (BPE). He had been treated with alfuzosin for the past two years. Rectal examination revealed an enlarged but smooth prostate. There was no history of contact with patient with tuberculosis.<br />\r\n	The patient&rsquo;s peripheral blood and renal profile were normal. Urine analysis showed presence of leucocytes (500/ul), pus cells (20-50), red blood cells (50/ul) and bacteria (1+), along with protein (0.25g/L) and glucose (3mmol/L). However, urine culture for mycobacterium tuberculosis was negative. Ultrasound revealed an enlarged prostate that measured 6 x 6 x 6 cm. Transurethral resection of the prostate was done, along with insertion of suprapubic tube. Histopathological examination of the prostate revealed tuberculous caseating granulomatous inflammation with benign prostatic hyperplasia (Figure 1). Ziehl Neelsen stain showed presence of acid fast bacilli, confirmed the diagnosis of tuberculosis (Figure 2).<br />\r\n	He was given anti-tuberculosis treatment which consisted of ethambutol, rifampicin, isoniazid and pyrazinamide, along with vitamin B12. Subsequently, further investigations were performed to look for any pulmonary involvement of TB. Chest X-ray was clear with no evidence of tuberculosis. Sputum examination for acid-fast bacilli was also negative. Erythrocyte sedimentation rate was elevated (81 mm/hr). Incidentally, urine culture showed growth of Klebsiella ESBL which was sensitive towards ertapenem.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	TB of the prostate is uncommon and the possible modes of involvement include descending infection from the urinary organs or hematogenous spread (Gupta et al. 2008). Based on clinical observations and animal experiments, Sporer and Auerbach (1978) suggested that TB of the prostate is almost always the result of one or perhaps repeated hematogenous seedings. Direct extension may occur (Sporer &amp; Auerbach 1987; Rai et al. 2009). The predisposing factors associated with the development of TB are related to poor immune status such as long-term use of steroid, the use of immunosuppressive drugs and diseases that impaired cell-mediated immunity (Gow 1998).<br />\r\n	The diagnosis could be difficult, if there was only a microscopic focus of granuloma in a large quantity of TURP prostatic sample. Furthermore, if occurring in an immunocompetent individual without any respiratory symptoms to suggest tuberculosis, it could be missed. Hence, this represents a diagnostic pitfall for histopathologists. Indeed, it is a differential diagnosis to be kept in mind, especially in endemic countries.<br />\r\n	Culture is the gold standard for diagnosis of genitourinary tuberculosis. Tissue and urine may be obtained for culture for mycobacterium tuberculosis. However, tissue samples are rarely sent for culture, unless there is a suspicion of tuberculosis. A study by Mortier et al. (1996) suggested that urine sampling for mycobacterium tuberculosis culture is useful for genitourinary tuberculosis exhibiting signs and symptoms of the disease. It is possible to perform Ziehl-Neelsen (ZN) staining of urine. However, the test requires certain amount of bacteria present in order to yield a positive result. It has a high specificity (100%) but low sensitivity (25%) (Ghaleb et al. 2013).<br />\r\n	It is also possible to perform polymerase chain reaction (PCR) using tissue and urine to detect tuberculosis. Study by Chawla et al. (2012) showed that tissue and urine PCR results are coinciding with histopathological results. Tissue and urine PCR have a sensitivity of 87.5% and a specificity of 86.7% with some variations. However, urine PCR may not be useful in isolated involvement of the prostate, penis and the seminal vesicles (Chawla et al. 2012).<br />\r\n	In conclusion, there are various ancillary tests that may be used to detect mycobacterium tuberculosis in order to establish its diagnosis. Culture and ZN stain are the most widely available tests offer by most laboratories, while PCR is technically more demanding, offers usually by laboratory with molecular diagnostic facility. Lastly, tuberculosis of the prostate is possible even in an immunocompetent individual. It should be a differential diagnosis in men who present with lower urinary tract symptoms, if they lived in areas where TB is endemic.</p>\r\n', 1),
(1124, 1116, '<p>\r\n	INTRODUCTION<br />\r\n	Orbital cellulitis is an aggressive infective process that affects the orbital adnexal structures posterior to the orbital septum. It should be differentiated from a more benign but common condition known as preseptal cellulitis (Chaudhry et al. 2008). Orbital cellulitis carries high morbidity and mortality between 20% to 40% (Duke-Elder &amp; Macfaul 1974; Maniglia et al. 1989). Early investigations that include imaging modality may greatly assist in its diagnosis and management. Once diagnosed, an aggressive surgical intervention couple with efficient medical therapy may help to improve the outcome.</p>\r\n<p>\r\n	<br />\r\n	CASE REPORT<br />\r\n	Ms NA, 15-year-old female, was brought by her friend to the Emergency Department (ED) of Hospital Melaka in February 2010 evening for unsettling high grade fever and headache associated with swelling over her right forehead. She was suffering from fever with rigors for four days which partially relieved with paracetamol. However, her right forehead swelling was increasing in size and started to discharge pus. There was also bilateral Malaysiaeye swelling that was associated with pain especially on movements of the right eye. She also had poor appetite and loose stools for two days with bowel movement of 4-5 times per day, watery in nature but not associated with blood stained. Previously in 2008, she was diagnosed with brain tumour but defaulted her treatment and follow-up. Apart from that, she had no other medical illness.<br />\r\n	Upon arrival at ED, she was conscious with Glasgow Coma Scale (GCS) of 15/15 but dehydrated. Her vitals were as follows: blood pressure of 110/70 mmHg, pulse rate of 140 bpm, temperature of 400C, respiratory rate of 16 breath per minute, and pain score of 3/10. Her capillary blood glucose (CBG) was 6.5mmol/L. Examination of her face was as per diagram shown (Figure 1). Other examination findings was unremarkable.<br />\r\n	Patient was triaged to semi-critical area and started with intravenous normal saline therapy at 100 ml/hr. She was given oral paracetamol of 1 gram and intravenous Cloxacillin 1 gm after swab for culture and sensitivity was taken from her forehead.<br />\r\n	Her blood investigations revealed elevated TWBC of 20.2 % (predominant neutrophils 90%), normal hemoglobin (12.6 gm/dl) and platelet of 168. Her renal profiles were as follows; Na+ of 123 mmol/l, K+ 3.4 mmol/l, urea of 2.3 and creatinine of 43. Her liver panel was within normal limit. Non-contrasted CT head showed hydrocephalus with suspicious lesion in midbrain which raised the possibilities of infection as well as tumour.<br />\r\n	Patient&rsquo;s GCS deteriorated to 9/15 (E1/V3/M5) the next day while in the ward. She was electively intubated in view of inability to protect airway and brought to ICU for further care. Ophthalmology team was called in and lateral canthotomy right eye was done under aseptic technique. MRI of brain was done and reported as oedema at facial and scalp in favour of cellulitis with no focal abscess collection within the tissue. Fluid collection at right retrobulbar space was suggestive of an abscess (Figure 2). Other findings included mild hydrocephalus, tumour in brainstem, left cerebellum and basal ganglia suggestive of glioma. Emergency drainage of abscess was done under general anasthesia (GA). However, the patient succumbed at day-10 of admission despite all efforts.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	Orbital cellulits is one of the life threatening event that should not be missed or lightly looked into. It is an infective process involving ocular adnexal structures posterior to the orbital septum and must be distinguished from preseptal cellulitis (sometimes called periorbital cellulitis), which is an infection of the anterior portion of the eyelid. Neither infection involves the globe itself. Although, preseptal and orbital cellulitis may be confused with one another because both can cause ocular pain and eyelid swelling and erythema, they have very different clinical implications. Preseptal cellulitis is generally a mild condition that rarely leads to serious complications, whereas orbital cellulitis may cause loss of vision and even loss of life.<br />\r\n	Orbital cellulitis is not common but can be devastating as highlighted in this case. It primarily affects children and adolescents between the ages of 0&ndash;15 years. It spreads to the orbit via three methods which are: (i) direct extension of infection from periorbital structure like paranasal sinuses also from face, the globe and the lacrimal sac (ii) haematogenous spread and (iii) direct inoculation into the orbit as result of trauma or surgery.<br />\r\n	In Paediatric age group up to 85% are due to complication of ethmoid sinusitis (Schramm et al. 1978). Therefore, it explains the fact why there is increased incidence in winter because of the increased incidence of sinusitis in cold weather. Suhaili et al. (2010) discovered six cases of orbital cellulitis while conducting a retrospective study for year 1997-2007 as complication of acute sinusitis in patient below 18 years of age in Universiti Kebangsaan Malaysia Hospital, Malaysia (Suhaili et al. 2010). Another earlier study in Universiti Malaya, Malaysia (2008) found that of 1198 patients in their registry with eye problems only one had orbital cellulitis making its prevalence of 0.1% (Reddy et al. 2008). In this patient, the orbital cellulitis was not caused by complication of sinusitis but instead occurred as direct inoculation of orbit as complication of acne that become an abscess at her right forehead. This infection later lead to septic shock as evidenced by clinical deterioration and blood culture that grew Staphylococcus aureus.<br />\r\n	The challenges remain in diagnosing orbital cellulitis as early treatment play crucial role in improving outcome, reducing morbidity and mortality rate. Prior to availability of antibiotics, the mortality rate was as high as 17%, while blindness rate at 20% (Williams &amp; Carruth 1992). Orbital cellulitis classically presents with ophthalmoplegia, pain with eye movements, proptosis and needs to be confirmed by radiological studies (Mair et al. 2002). In this patient, the CT head was done the next day when she deteriorated clinically and opthalmology referral was delayed as well. This highlights the need of high index of suspicion in order to reach the diagnosis of orbital cellulitis eventhough patient did not has the classical or typical complaints. Mair et al. in 2002 proposed the use of ultrasound as aid to diagnose and monitor treatment of acute swelling of eyelids in Paediactric age group patients (Chaudhry et al. 2012). Through this research, orbital sonography is recommended in every children with periorbital swelling and erythema as it can detect orbital infection or at least prompt further investigations avoiding delay in treatment and allows disease monitoring on a daily basis.</p>\r\n<p>\r\n	CONCLUSION<br />\r\n	Orbital cellulitis is an eye emergency that requires high index of suspicion in order not to be misdiagnosed. Once suspected, urgent referral is warranted. Radiological imaging is needed in order to confirm this diagnosis and ultrasound can play essential role in differentiation between pre and postseptal infection. Aggressive early medical and surgical treatment is needed in order to save life.</p>\r\n', 1),
(1129, 1121, '<p>\r\n	INTRODUCTION<br />\r\n	Eosinophilic gastroenteritis (EG) is a rare manifestation of HES with a frequency of around 25% (Rothenberg 2004). The clinical features of EG varies is based on the region and depth of the gastrointestinal tract (GIT) affected by the eosinophilic infiltration (Rothenberg 2004; Shifflet et al. 2009; Freeman 2008; Conus &amp; Simon 2008). The common symptoms include abdominal pain, diarrhea and vomiting.The definitive diagnosis of EG requires histological evidence of eosinophilic infiltration of &gt;20 eosinophils/high-powered field in the lamina propria (Rothenberg 2004; Shifflet et al. 2009; Liacouras 2007). The main aim of the treatment is to reduce the eosinophil counts. In general, the majority of patients respond well to steroid therapy (Conus &amp; Simon 2008; Liacouras 2007; Chen et al. 2003). However, relapses are common and such patients may require long term immunosuppressive agents (Chen et al. 2003).</p>\r\n<p>\r\n	CASE REPORT<br />\r\n	A 22-year-old lady with a past medical history of well controlled eczema and asthma presented with a history of diarrhoea, vomiting and abdominal pain for one week. The initial blood investigations showed leucocytosis with marked eosinophilia (TWC of 41.2 with eosinophil of 69%) (Figure 1). She was treated initially as acute gastroenteritis with intravenous fluid and antibiotics but her symptoms persisted. Over the span of few days, she developed dyspnoea. Her chest radiograph and CT scan of her chest and abdomen revealed bilateral pleural effusion and ascites. She also developed purpuric rashes over the dorsum of her hands associated with joint pain.<br />\r\n	Her blood, stool and urine cultures were negative. Stool specimens were also negative for ova and cysts. In view of her persistent diarrhoea, patient underwent an upper and lower gastrointestinal endoscopy. Her upper endoscopy showed pangastritis and duodenitis. The gastric and duodenal biopsies showed significant oeosinophil infiltration (&gt;20 per high power field) each (Figure 2). Her colonoscopy revealed mild ileitis and pancolitis predominantly involving the descending and sigmoid colon. The biopsies taken throughout the colon and terminal ileum also demonstrated presence of mild to moderate infiltration of the lamina propia by eosinophils of &gt;30 per high power field (Figure 3 and 4). A skin biopsy revealed scattered eosinophils at the periadnexal and perivascular area. Bone marrow trephine exhibited increased number of eosinophils and its precursors. No malignant cells were observed.</p>\r\n<p>\r\n	Hence, the provisional diagnosis was idiopathic hypereosinophilic syndrome with oeosinophilic gastroenteritis, cutaneous involvement and serositis. The patient was started on prednisolone 0.5mg/kg and the dose was tapered slowly over the next four weeks. She was discharged well and six weeks after treatment, she was asymptomatic and her serositis had resolved. Her total white count normalized and the percentage of oeosinophils reduced to 9%.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	Idiopathic hypereosinophilic syndrome (HES) constitutes a rare and heterogeneous group of disorders with unknown prevalence (Roufosse et al. 2007). The term HES was first introduced by Hardy and Anderson in 1968 and in 1975, Chusid et al established four diagnostic criteria for idiopathic HES (Roufosse et al. 2007; Kahn et al. 2008; Klion 2005) namely the presence of i) blood eosinophilia &gt;1500/mm3 for at least six months, ii) absence of an underlying cause of oeosinophilia despite extensive evaluation, and iii) presence of end organ damage or dysfunction related to the oeosinophilia. HES has a predilection for the skin, heart and nervous system but almost any tissue or organ can be affected. Of note, the cardiovascular and hematological systems account for more than 50% of the disease manifestation (Roufosse et al. 2007). The onset of HES is generally between the age of 20-50 years with male preponderance (Freeman 2008; Klion 2005).<br />\r\n	About 25% of HES patients have oeosinophilic infiltration of the gastrointestinal tract which may involve almost any region from the esophagus to rectum (Freeman 2008). Although the oeosinophilic infiltration in EG can involve any part of the gastrointestinal tract, this disease is characterized by predominant involvement of the gastric and small intestinal mucosa (Rothenberg 2004). EG as the initial manifestation of HES with such extensive involvement of the entire digestive tract is extremely rare.<br />\r\n	The exact etiology of EG is unknown but up to 40% cases were associated with underlying allergies or atopy namely food allergies, eczema, allergic rhinitis and asthma (Chen et al. 2003; Nair et al. 2011). The elevated Ig E levels in the sera of affected patients implies that atopy maybe involved in the pathogenesis of EG (Rothenberg 2004; Chen et al. 2003). In the present case, the patient had asthma and eczema.<br />\r\n	Iron deficiency associated with blood loss as well as protein-losing enteropathy are known complications of this disease entity. If muscular layers are involved, intestinal obstruction may ensue, while serosal involvement may be associated with ascites (Rithenberg 2004; Freeman 2008; Chen et al. 2003).<br />\r\n	The endoscopic appearance of EG is nonspecific. The abnormal findings include erythematous mucosa, friable, nodular, and ulcerative changes. In a retrospective study by Chen et al. (2003) involving 15 patients with EG, 10 of the patients had only nonspecific gastritis or colitis while two patients had shallow gastritis and duodenal ulcers (Chen et al. 2003).<br />\r\n	Definitive diagnosis requires histological evidence of eosinophilic infiltration. Oeosinophilic infiltration is usually patchy in distribution and may be present in otherwise normal, non-inflamed bowel wall (Rothenberg 2004; Chen et al. 2003). Because of this, multiple biopsies should be taken to improve diagnostic yield. For the purpose of deriving the diagnosis of EG, the majority of studies have used a definition of &gt;20 oeosinophils/ high-powered field in the lamina propria (Rothenberg 2004; Shifflet et al. 2009; Liacouras 2007).<br />\r\n	The evidence for the management of EG is largely based on case studies and retrospective studies (Liacouras 2007; Chen et al. 2003). Presently, there is no randomized trial on its treatment because of the rarity of the disease (Liacouras 2007). The main aim of the management of EG is to reduce the oeosinophil counts. In general, patients respond rapidly to steroids (Conus &amp; Simon 2008; Liacouras 2007; Chen et al. 2003). However, the optimal duration of the steroid therapy is unknown but Chen et al. (2003) demonstrated that short courses of steroid therapy followed by repeated courses for relapses gave promising results in treating EG (Chen et al. 2003). Our patient was treated with prednisolone 0.5 mg per kg for two weeks which was tapered after two weeks. This regime resulted in marked symptomatic and radiological improvement. For patients who have frequent relapses of EG, long term immunosuppressive agents or low dose corticosteroids is strongly recommended (Chen et al. 2003). Few authors have described the use of sodium cromoglycate (a stabilizer of mast cell membranes) or montelukast (a selective, competitive leukotriene receptor antagonist) as steroid-sparing agents (Schwartz et al. 2001).</p>\r\n', 1),
(1133, 1125, '<p>\r\n	INTRODUCTION<br />\r\n	The coeliac trunk (CT) is the most cephalic unpaired ventral branch of abdominal aorta. It commonly divides into left gastric, common hepatic (CHA) and splenic arteries. The superior mesenteric artery (SMA) is the second ventral branch of abdominal aorta. The branches of SMA are inferior pancreaticoduodenal, jejunal, ileal, ileocolic, right colic and middle colic arteries. The middle colic artery (MCA) descends in the transverse mesocolon and divides into right &amp; left branches (Standring 2005). These branches form part of marginal artery and supply mostly transverse colon.<br />\r\n	Variations in the branching pattern of ventral branches of abdominal aorta are not infrequent. There have been reports of anomalies of coeliac trunk in the past. The common hepatic artery originates from coeliac trunk in 51-80% cases (Adachi 1928; Chen et al. 1998). Several other branches such as inferior phrenic arteries, dorsal pancreatic artery and middle colic artery may also arise from the coeliac trunk (Chitra 2010). Aberrant origin of middle colic artery have been reported earlier (Yildrim et al. 2004). Adachi (1928) observed origin of middle colic artery from the coeliac trunk in 0.5% to 1% cases. Middle colic artery originating from the proximal segment of splenic artery as observed in the present case is a rare anomaly. A precise knowledge of such arterial variations are important for diagnostic procedures and surgeries of upper abdomen.</p>\r\n<p>\r\n	CASE REPORT<br />\r\n	During routine dissection of abdominal retroperitoneal region for first year medical students, a variant branching pattern of ventral branches of abdominal aorta was detected in a 50-year-old male cadaver. The specimen was photographed after delineating the surrounding structures.<br />\r\n	The coeliac trunk originated from the ventral aspect of abdominal aorta and bifurcated into left gastric and splenic arteries. The common hepatic artery arose directly from the abdominal aorta 0.2 cm inferior to the origin of celiac trunk. Nothing abnormal was observed in the further course and branching pattern of common hepatic artery.<br />\r\n	The middle colic artery, normally a branch of superior mesenteric artery originated from the proximal segment of the splenic artery (Figure 1). The artery then passed posterior to the body of pancreas to enter into transverse mesocolon (Figure 2). Two cms proximal to the transverse colon, it divided in to right and left branches. These branches anastomosed with the ascending branch of iliocolic and left colic arteries respectively to form the marginal artery. The length of the middle colic artery was 12 cm and had a diameter of 0.8 cm, near its origin. It gradually narrowed towards its termination. No separate middle colic artery originating from superior mesenteric artery was seen. Other branches of superior mesenteric artery i.e. right colic, ileocolic, ileal and jejunal branches were normal. The distance between the coeliac trunk and the superior mesenteric artery was 1 cm.</p>\r\n<p>\r\n	<br />\r\n	DISCUSSION<br />\r\n	Variations of hepatic artery are important in liver transplantation. There have been reports of right hepatic artery originating from the middle colic artery, directly from the aorta and a left hepatic artery originating from the common hepatic artery (Ugurel et al. 2010). The origin of hepatic artery are of six types according to Hiatt&rsquo;+s clasiification. The origin of CHA directly from the aorta belongs to type VI (Hiatt et al. 1994). The present case also observed the type VI origin of CHA.<br />\r\n	The superior mesenteric artery or one of its major branches give origin to middle colic artery. It may arise from the (a) coeliac trunk (Standring 2005), (b) abdominal aorta between the origin of superior and inferior mesenteric artery (Benton &amp; Cotter 1963), (c) inferior mesenteric artery (Benton &amp; Cotter 1963; Chen et al. 1998) , (d) common hepatic artery (Cavdar et al. 1998). It may be absent in 3 to 5% of cases (Hiatt et al. 1994). However, the origin of middle colic artery from the proximal segment of splenic artery is a rare anomaly and to the best of our knowledge only one case has been reported in the past (Chou et al. 1997). The unusual origin, variable course and a smaller lumen of the anomalous middle colic artery increases the risk of vascular damage to ascending and transverse colon.<br />\r\n	The variations of the coeliac trunk can be due to abnormal embryological development of the ventral splanchnic arteries. The anomalous middle colic artery originating from splenic artery can be due to the persistence of longitudinal anastomosis between the vitelline arteries in the embryo (Cavdar et al. 1998).<br />\r\n	The origin of common hepatic artery directly from abdominal aorta and middle colic artery from the splenic artery is a rare finding and such a possibility should be kept in mind to ensure a safe and successful abdominal surgery.</p>\r\n<p>\r\n	CONCLUSION<br />\r\n	Awareness of anatomical variations in the origin and branching pattern of arteries arising from the coeliac trunk and superior mesenteric trunk are necessary for surgeons and radiologist to avoid vascular complications.</p>\r\n', 1),
(1139, 1131, NULL, NULL),
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(1166, 1158, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Bone is a highly metabolic tissue where it functioning as providing balance to the body, protect the inner organs and also play a role as calcium storage. It is a compound of hydroxyapatite (45%), type I collagen (40%), and water (15%). Structure level of cortical bone and trabecular bone are differ (Eckstein et al. 2007). Bone continuously models and remodels. In the cellular level, the modeling and remodeling process are not very differing. Because it is based on the separate actions of bone resorbing cells that are called osteoclasts, while bone forming cells is called osteoblasts. Bone formation exceeds bone remodeling in young individuals where the peak bone mass (PBM) is reached around the age of 20-30 years (Hara et al. 2001; Kohrt et al. 2004). The remodeling development is influenced by hormones where when the person become ageing, the level of estrogen and testosterone decrease, and will decrease bone remodeling for bone formation. In adults, peak bone mass, rate and amount of bone loss is a determinant of bone mass (Eckstein et al. 2007).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	<div>\r\n		THE RISING INCIDENCE OF OSTEOPOROSIS</div>\r\n	<div>\r\n		Osteoporosis is known as a silent disease because bone density slowly decrease with advancing age and without symptoms (Naina Mohamed et al. 2012). Osteoporosis is a progressive skeletal disease that can be described by weakening of bone and low bone density andwhich becomes clinically obvious when there is a fracture (Mcclung 2003). There are two major risk factors for osteoporotic fractures which are age and bone mineral density (BMD) (Hui et al. 1988). Osteoporosis in developing countries is on the rise (Handa and Kalla 2008). This disease causes major problems in terms of the cost to society and the cost to suffering individuals where it results in a significant increase in healthcare cost (Loh &amp; Shong 2007). Every year, the incidence of osteoporosis and osteoporosis fracture rise with an aging society. In 1996, the elderly population (more than 50 years old) in Malaysia was approximately 2.45 million (Lee &amp; Khir 2007) and it increased to 4.52 million in 2010 (Department of Statistics 2010). In 1997, the cost for hospitalizationbecause of hip fracture was estimated at Ringgit Malaysia 22 million, with high individual cost which many could not afford (Malaysia Osteoporosis Society 2006). Osteoporosis is one of the most important public health issues that affects the elderly in many countries, including developing countries like Malaysia.</div>\r\n	<div>\r\n		In Malaysia, the incidence of hip fracture among individuals over 50 years of age is 0.9/1000 individuals with the highest being in Chinese women (Malaysia Osteoporosis Society 2001). Ethnicity is one of the factor to determine and influence BMD. Black and Asian women showed low BMD within each age group (Handa &amp; Kalla 2008). It is expected that the incidence of osteoporosis is increasing rapidly in ageing population. Statistics in Malaysia showed that the percentage of population aged above 65 years grows at the rate of 3% yearly and by the year 2020 there would be 7.3% or 2 million elderly people in the country (Department of Statistic 2010).</div>\r\n	<div>\r\n		According to the criteria set by the World Health Organization, an individual is diagnosed as osteoporotic if his or her BMD is 2.5 standard deviations (SDs) below that of a healthy young adult of the reference population or more commonly expressed as a T-score of -2.5 or less (World Health Organization 1994). Several parts or a site in our body are associated with fragility fractures which includes the hip, wrist, vertebra and rib (Yeap et al. 2012). Based on the report by the World Health Organization, 2004, it is estimated that 4.5 million people suffered from an osteoporotic fracture in America and Europe. Table 1 shows the estimated number of osteoporotic fractures by site in men and women with age more than 50 years in 2000 by the World Health Organization (World Health Organization 2004).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		BONE HEALTH SCREENING FOR EARLY DETECTIONAND PREVENTION OF OSTEOPOROSIS</div>\r\n	<div>\r\n		In order to prevent the progression to osteoporosis, early detection of bone density loss is very important (Laugier 2004). Bone densitometry is currentlyused to classify individuals at greatest risk of fracture and to diagnose osteoporosis so that they can be informed that they have osteoporosis and early treatment can be initiated (Kanis &amp; Gl&uuml;er 2000). Bone loss can be assessed by several bone-densitometry methods; Dual-energy x-ray absorptiometry (DXA), quantitative computed tomography (CT), peripheral quantitative computed tomography (QCT), magnetic resonance imaging of plain films (Radiogrammetry), and quantitative ultrasound (QUS). Each tool provides distinct advantages and disadvantages which depend on the purpose or outcome of its application (Bachrach &amp; Sills 2011).</div>\r\n	<div>\r\n		The diagnosis of osteoporosis depends on the assessment of two bone properties. Those are bone quantity with bone mass expressed as bone mineral density (BMD) and bone quality reflectto micro-architecture properties of the bone. Bone mineral density (BMD) is the main clinical assessment to assess bone mass. Therefore, measurement of bone mass using bone densitometry form the basis for the diagnosis of osteoporosis (Kanis et al. 1997). Bone mineral density (BMD) is defined as an index of bone mass derived from the amount of mineral measured per unit area or volume of bone tissue (Kanis 2002).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		BONE DENSITOMETRY: QUS AS AN ALTERNATIVE TO DXA</div>\r\n	<div>\r\n		In measuring bone mineral density (BMD), the current gold standard for detecting osteoporosis is using dual-energy X-ray absorptiometry (DXA) (Laugier 2004). However, there are anumber of disadvantages in the use of the DXA, which include lack of the equipment, high cost, importability, and ionizing production (Laugier 2004) which limits its application for large population bone health screening programmed.</div>\r\n	<div>\r\n		There is a wide growing interest in the use of quantitative ultrasound (QUS) amongst various technologies, for screening purposes. Quantitative ultrasound (QUS) of bone was introduced approximately two decades (1984) ago as a device for examining bone tissue (Laugier 2004) and as potential technologies in managing osteoporosis. This new health technology has attracted extensive scientific and clinical interestwhere Quantitative ultrasound (QUS) is a new method that is free from radiation, non-invasive, portable and cheaper compare to Dual X-ray Absorptiometry (DXA) (Laugier 2004). QUS scanners gives benefits to patients and doctors in the assessment of osteoporosis (Kanis &amp; Gl&uuml;er 2000). Quantitative ultrasound (QUS) evaluation of bone health status has gained popularity in recent years, especially in regions with limited access to dual X-ray absorptiometry (DXA) device. As an alternative to DXA for osteoporosis screening, quantitative ultrasound technology has gained acceptance and is currently being investigated as a tool that can replace DXA (Chin et al. 2012). Quantitative ultrasound is a useful tool for a clinical study which gives useful information about bone status and very effective in the management of patients (Guglielmi &amp; Terlizzi 2009).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		TECHNICAL ASPECTS OF QUS</div>\r\n	<div>\r\n		There are two types of QUS, (Chin &amp; Ima-Nirwana 2013) depending on the type of the ultrasound transmission travelling through the bone. First is axial or horizontal transmission of the ultrasound waves which travel along the cortical layer of the phalanges, radius or tibia segment of the bone. Second is the transverse or longitudinal transmission of the ultrasound waves which travel either through trabecular bone or cortical bone.</div>\r\n	<div>\r\n		According to official International Society for Clinical Densitometry (ISCD) in 2007, the committee stated that for clinical use of QUS, calcaneal is the only validated skeletal site for the management of osteoporosis (Krieg et al. 2008). Prospective study was conducted to validate QUS against DXA for ability to predict osteoporosis fracture risk and results demonstrated that calcaneal QUS by transverse transmission devices predicts fracture accurately and better compared toother QUS devices at other skeletal sites such as the cortical phalange, and cortical radius devices (Krieg et al. 2008). Trabecular bone can be found highly at calcaneal site while some other sites of measurement are primarily cortical. The coefficient of variation can appear particularly good for cortical sites due to the higher speed of sound (SOS) in cortical bone compared to trabecular bone when measuring the SOS. It can be concluded that calcaneus QUS measurement is able to predict hip fractures and all osteoporotic fractures in elderly woman and man with similar performance to hip DXA measurements (Stewart et al. 2006). Besides, the calcaneus possesses two lateral surfaces and consists of 95% trabecular bone, which helps the movement of ultrasound through it where it is easily accessible. Furthermore, calcaneum has medial and lateral aspects being relatively flat and parallel, with a high metabolic turnover rate and a pattern of bone loss similar to the spine (Knapp 2009).</div>\r\n	<div>\r\n		There are several parameters generated by QUS. The panel from International Society of Clinical Densitometry (ISCD) has rated the following indices as appropriate terminology (Krieg et al. 2008) and as the accepted outcome measures for calcaneal QUS devices:</div>\r\n	<div>\r\n		1. Recommended attenuation parameter: Broadband Ultrasound Attenuation (BUA) expressed in dB/MHz,</div>\r\n	<div>\r\n		2. Recommended velocity parameter: Speed of Sound (SOS) expressed in meters per second (m/s),</div>\r\n	<div>\r\n		3. Composite parameter: Stiffness Index (SI) or Quantitative Ultrasound Index (QUI) is generated by combining BUA and SOS. These two composite parameters may be clinically useful in the determination of subjects having low bone health status (Chin &amp; Ima-Nirwana 2013).</div>\r\n	<div>\r\n		The speed of sound (SOS) refers to the division of transmission time of the sound waves by the length of the body part studied. Unit used in the SOS measurement is meter per second (m/s). Broadband attenuation (BUA) of sound refers to the slope between attenuation of sound signals and its frequency, The unit used is dB/MHz. Soft tissue and bone absorbs energy when the sound wave undergo them, thus attenuation occurs. In recent times, latest QUS indices derived from at least two basic measurements such as amplitude-depend SOS (AD-SOS), stiffness index (SI), quantitative ultrasound index (QUI) and estimated BMD (eBMD) (Chin &amp; Ima-Nirwana 2013). Several previous study in the past have examined the usefulness of QUS and its potential role in the field of osteoporosis whereboth broadband ultrasound attenuation (BUA) and speed of sound (SOS) measurements at the calcaneus can be used to identify individualswho are at risk of osteoporotic fracture as reliably as BMD (Bauer et al. 1997; Frost et al. 2001; Hans et al. 1996).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		THE APPLICATION OF QUANTITATIVE ULTRASOUND (QUS)</div>\r\n	<div>\r\n		The WHO T-score diagnostic classification derived from DXA measurement either at spine, hip or forearm cannot be applied to T-score derived from non-DXA measurements such as QUS because both measurements employs different technology in assessing individuals bone health status. Furthermore, it is impossible to apply the WHO T-score to other skeletal sites without having significant discrepancies.However, previous studies have demonstrated significant correlation between calcaneal QUS parameters with BMD (Dane et al. 2008; T&ouml;yr&auml;s et al. 2002) and skeletal site-matched bone mass assessed by DXA or peripheral DXA (Krieg et al. 2008). Subsequently, one could classify individuals into respective risk groups (the likelihood of having osteoporosis) by calculating the specific upper and lower threshold concept to QUS. Unlike DXA which centre its assessment only on bone mass, QUS not only measures bone mass but also measures other bone properties of bone strength which reflect bone micro architecture and material properties (Laugier 2004) (Trimpou et al. 2010) (Stewart et al. 2006). QUS is affordable, inexpensive, widely available, easy, fast, and transportable and most importantly does not produce any radiation, thus suitable to be used for large epidemiological bone health study.</div>\r\n	<div>\r\n		Two meta-analysis on fracture risk assessment using QUS by Marin and Moayerri (Marin et al. 2006) (Moayyeri et al. 2012) provide a concrete conclusion that QUS parameters are able to predict fracture risk as good as central DXA. Previous studies have demonstrated that assessment of QUS at the calcaneal site is able to predict fracture in postmenopausal women (Kanis 2002). The ability of QUS to predict fracture was also proven in elderly men and QUS has the capability to discriminate between male and female subjects (Chin et al. 2013) at various skeletal sites and independently of the BMD assessed by central DXA. Generally, QUS has the ability to discriminate those with osteoporotic fractures (fracture at either hip, spine or any osteoporotic fractures) and those without osteoporotic fracture (Krieg et al. 2008). Furthermore, QUS overcomes the limitation by offering an opportunity for bone health assessment particularly to people without access to DXA scanning. It is also highly suitable for use in fragile populations such as children, pregnant mothers and remote access populations.</div>\r\n	<div>\r\n		Several studies have proved that calcaneal trabecular transmission QUS parameters are highly associated with BMD measured by DXA In human studies, SOS was found to be significantly correlated with BMD at various skeletal sites as assessed by Dane and colleagues in a cross-sectional study in 2008 (Dane et al. 2008) and by monitoring the changes of DXA and QUS at first screening and during the seven year follow-up period in a longitudinal study by Trimpou et al. in 2010. Therefore, from these two studies, it can be concluded that QUS parameters positively correlates with both BMD and BMC measured using the DXA machine. Thus, long term precisions by QUS need to be established by several large human cohort studies for the purpose of generalizing heel QUS in monitoring treatments.</div>\r\n	<div>\r\n		BUA and SOS measurements gives indication for osteoporosis, and. Therefore, calcaneal QUS could become an alternative tool in identifying individuals who have low bone mass and possible candidates for therapeutic intervention (Frost et al. 2001). The precision of QUS is high, which make the device to be used in epidemiological studies (Trimpou et al. 2010). Some practitioners use the QUS as an indicator of risk of fracture as for example individuals who are at high risk, medium risk, or low risk, where it is dependent on the QUS parameters and also the presence of strong clinical risk factors (Stewart et al. 2006). Risk of osteoporotic fractures also can be identified in the community in postmenopausal women, as an improved process of targeting women for axial DXA BMD measurement to diagnose osteoporosis, with care, to target antiresorptive treatments when low QUS measurements are present, in addition to major clinical risk factors (Stewart &amp; Reid 2002).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		QUANTITATIVE ULTRASOUND (QUS) AND MEASUREMENT OF BONE PROPERTIES</div>\r\n	<div>\r\n		QUS is capable in measuring micro-architectural and the density of bones (Gluer et al. 1994; Njeh et al. 2001). In-vitro studies shows that the ultrasound derived modulus of elasticity and correlates strongly with values of bone breaking strength derived from static loading (Turner &amp; Eich 1991). In addition, a study reported that QUS parameters are significantly associated with bone structure independently of BMD (Gluer et al. 1994). As measured by mechanical testing, Mehta et al. reported ultrasound velocity and elasticity strongly connected with material elasticity (Mehta et al. 1998). Broadband ultrasound attenuation (BUA) values showed to be dependent upon trabecular orientation in vitro (Gluer et al. 1994). However, using QUS and DXA in-vivo, there was high correlation of r=0.75 to r=0.90 between BUA and BMD at the calcaneus with matched regions of interest, suggesting that QUS may reflect micro-architecture. Njeh et al. (2001) in a review of whether QUS is dependent on structure concluded that ultrasound attenuation is due to structural parameters as well as dependent on density (Njeh et al. (2001).</div>\r\n	<div>\r\n		The much strength of QUS does not exempt it from weaknesses. Different QUS devices made by different manufacturers employ diverse technologies and thus, different devices from different producer have different indices calculation algorithms. This is further complicated by difference in types of model, different skeletal sites of measurements and analysis and different calibration methods. As a result, bone density measurements by QUS from different manufactures and models will have significant differences and therefore cannot be directly compared. Comparison to various DXA devices with different manufactures and models is also not possible and cannot be performed due to differences in their calculation even though all DXA instruments use absolute BMD values as the common outcome measure (Krieg et al. 2008). In terms of precision, QUS has less precision when compared to DXA (Ravaud et al. 1999). Broadband ultrasound attenuation precision also appears to be poorer than its corresponding SOS precision in the same device. As a result of the poor precision of QUS in comparison to DXA, optimization of measurements to reduce precision errors is of greatest importance (Knapp 2009).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		CONCLUSION</div>\r\n	<div>\r\n		To conclude, QUS is sensitive to age and menopause-related changes, clinical risk factors and lifestyle factors related to osteoporosis. QUS and DXA have been shown to be predictive of osteoporotic fractures, especially in the elderly. QUS has been widely researched upon and has been demonstrated to have the capability to predict fracture, particularly at the hip. QUS also can be used as a screening tool for bone health, for assessment of risk of fracture in the community in postmenopausal women, as an improved method of targeting women for axial DXA BMD measurement to diagnose osteoporosis, and also to target antiresorptive treatments when low QUS measurements are present in addition to major clinical risk factors (Stewart &amp; Reid 2002).</div>\r\n	<div>\r\n		&nbsp;</div>\r\n	<div>\r\n		ACKNOWLEDGMENT</div>\r\n	<div>\r\n		The authors thank The Malaysia Bones Health and Osteoporosis Study (MALBONES) group and Mrs Nur Farahana Mohd Fozi from Department of Pharmacology, Universiti Kebangsaan Malaysia.</div>\r\n</div>\r\n', 1);
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(1172, 1164, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the common cause of neonatal jaundice in Malaysia. Neonatal screening for G6PD deficiency in Malaysia using the FST has long been established since 1980. The fallback of screening G6PD deficiency by this method is that semiquantitative test is not reliable in detecting female heterozygotes whereby they may haveFSTenzyme deficiency ranging from 20% to 60% of the normal residual activity. Detection of partially deficient G6PD individuals is important as they too are prone to develop acute hemolysis (Luzzatto &amp; Mehta 1995).</div>\r\n<div>\r\n	In a study conducted by Reclos et al. (2000) involving 2000 neonates, it was found that FST was able to detect all male neonates with severe G6PD deficiency in which the G6PD activitywas less than 2.1 U/gHb (less than 20% of the normal residual activity). However, FST misclassified almost all cases with G6PD enzyme activity between 20- 60% of the normal residual which was interpreted as normal FST. These cases were found to be exclusively in females. These findings were in concordance with a local study by Ainoon et al. (2003) involving 976 neonates. The study demonstrated that 46.3% of the cases with partial G6PD deficiency were misclassified as normal by FST with enzyme activity ranging from 2.92 to 8.7 U/gHb. They were found to be exclusively females, as well. Both studies emphasized on the importance of correct classification and proposed the employment of fully quantitative method as a screening tool, in which the partially G6PD deficient patients are also at risk of developing acute haemolytic crises similar to their male counterparts.</div>\r\n<div>\r\n	The OSMMR2000-D G6PD assay is a quantitative enzyme assay. This kit offers rapid, fully automated, quantitative measurement of G6PD coupled to a simultaneous evaluation of the haemoglobin (Hb) content in the sample, expressing the results in Units/gram haemoglobin (U/gHb). Hb normalization will allow for correction of all samples against a selected standard or control used in the test by &lsquo;normalizing&rsquo; all analyte values so that they correspond to the same quantity of sample (Azma et al. 2010) had established the normal reference range for G6PD enzyme activity level in Malaysian neonates and adults using this kit. The overall mean value for neonate and adult population were 12.43&plusmn;2.28 U/gHb and 9.21 U/gHb respectively. The normal reference range for neonate population was 10.15-14.71 U/gHb while the adult population was lower, between 6.61-11.81 U/gHb. In this study, there were no significant differences in mean G6PD activity between males and females for Chinese and Malays. This lack of difference in red cell G6PD activity level among ethnic and gender groups has been reported previously in Malays and Chinese in Malaysia using Sigma kit (Ainoon et al. 2003). Measurement of G6PD activity using OSMMR-D kit is slightly more expensive and laborious compare to FST. However, this test is far more sensitive in detecting partial G6PD individuals (Azma et al. 2010).</div>\r\n<div>\r\n	In this study, we established the normal range of G6PD activity using OSMMR-D kit with haemoglobin (Hb) normalization for paediatric population age one month to 12 years old. We also determined the prevalence of partial G6PD deficient in this population and normal term female neonates using similar enzyme assay kit and compared it with FST.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	A total of 236 subjects from paediatric population aged between 1 month- 12 years, who received treatment at Universiti Kebangsaan Malaysia Medical Centre (UKMMC) and 614 term female neonates born in UKMMC were enrolled in this study. The patients&rsquo; peripheral blood samples and cord blood in EDTA tube were sent to the Haematology Unit, Department of Diagnostic Laboratory Services, UKMMC.</div>\r\n<div>\r\n	For the establishment of normal range, selected paediatric blood samples were tested for full blood count using fully automated haematology analyzer (Coulter&reg;GENS). Those with normal haemoglobin level were included in this study.</div>\r\n<div>\r\n	For FST, samples were applied to Whatman&rsquo;s filter paper and dried at room temperature. The test was carried out according to the method described by Beutler and Mitchell (1968). The spot was examined for fluorescence production from reduced pyridine nucleotide (NADPH) under a long-wave ultraviolet light source. Those with normal FST were included in the determination of normal range for G6PD activity and cut-off points for partial G6PD deficiency. Those showed minimal G6PD activity with FST were included in the prevalence of partial G6PD deficiency study.</div>\r\n<div>\r\n	For the quantitation of G6PD activity, we used the OSMMR2000-D G6PD assay kit with haemoglobin normalization from R&amp;D Diagnostics (Holargos, Greece). The measurement of enzyme activity was done using a spectrophotometer (Ultramicroplate Reader EL808, Bio Tek, Instruments). Method for quantitation of G6PD activity using this assay kit has been described in a previous study (Azma et al. 2010).</div>\r\n<div>\r\n	Normal probability plots for variables of normal paediatric samples in each subgroup by age, sex and race were carried out to ascertain that the distribution was normal. Analysis of variance for the level of G6PD enzyme activity by sex, ethnic origin and age groups was carried out. This was followed by comparison of means in G6PD assay values between sex, ethnic origin and different age groups. A result of p&lt;0.05 was considered as statistically significant. Then the means, SD, ranges of G6PD activity for this paediatric group were determined. A 20% and 60% cut-off point for the diagnosis of severe and partial G6PD deficiency respectively, were also established in this group (WHO Working Group 1989).</div>\r\n<div>\r\n	For female neonates group, their cord blood samples were sent for FST analysis and quantitation of G6PD activity by OSMMR-D G6PD assay with Hb normalization. Prevalence of partial G6PD deficiency was determined in both paediatric and female neonatal group and findings were compared between the two methods.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	NORMAL MEAN OF G6PD ACTIVITY IN CHILDREN AGED 1 MONTH TO 12 YEARS OLD</div>\r\n<div>\r\n	From 236 paediatric subjects, aged between 1 month-12 years, only 214 subjects were included for the determination of normal reference range for G6PD activity. The other 22 subjects had absent or minimal enzyme activity by FST and G6PD enzyme assay showed G6PD activity less than 6.5U/gHb. They were considered as outliers and were excluded from the analysis. Out of 214 subjects, 117 were males (54.7%) and 97 were females (45.3%). Majority of the subjects recruited were Malays with 66.1% (n=156), followed by Chinese 18% (n=43), Indians 2.5% (n=6) and others 3.8% (n=9). There were no significant differences in the mean normal G6PD activity among age groups (Table 1). There were also no significant differences in the mean normal G6PD activity among races (Table 2), and among gender in each racial group (Table 3).</div>\r\n<div>\r\n	The overall mean value for normal G6PD activity was 10.18&plusmn;3.36 U/gHb (2SD). The reference range for normal G6PD activity established from this population was 6.82-13.54 U/gHb. The upper and lower limit cut-off points for partial G6PD deficiency were 6.11 U/gHb (60% of the normal mean) and 2.03 U/gHb (20% of normal mean), respectively (as recommended by WHO Working Group (1989). The upper limit for total/severe deficiency was 2.03 U/gHb (20% of the normal mean G6PD activity for paediatric population). The intrabatch and interbatch coefficient of variance for samples with normal G6PD enzyme activity level was 4.6%.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	PREVALENCE OF PARTIAL G6PD DEFICIENCY IN CHILDREN AGED 1 MONTH TO 12 YEARS OLD IN UKMMC</div>\r\n<div>\r\n	The analysis for prevalence of partial G6PD deficiency by FST and enzyme assay was based on 236 samples. The results of screening for partial G6PD deficiency by FST and OSMMR-D kit were shown in Table 4. In male paediatric group, none of them were found to be G6PD partially deficient either by FST or enzyme assay. There was one out of 115 cases (0.9%) in female paediatric group which showed minimal enzyme activity by FST, while 18 cases (15.7%) were identified as partial G6PD deficiency by quantitative enzyme assay. Therefore, only one case of partial G6PD deficiency was detected by routine FST and was later confirmed with enzyme assay. The other 17 (14.8%) cases which had normal FST were not detected by this method. These children with partial G6PD deficiency had enzyme activity level ranged from 4.23-5.26 U/gHb.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	PREVALENCE OF PARTIAL G6PD DEFICIENCY AMONG FEMALE NEONATES BORN IN UKMMC</div>\r\n<div>\r\n	A total of 614 term female neonates (459 Malays, 124 Chinese, 13 Indians and 18 others) were enrolled in this study. The overall mean normal G6PD activity and cut-off points for partial G6PD deficiency used in this study were based on study done by Azma et al. (2010).</div>\r\n<div>\r\n	For this study, 21 (3.42%) female neonates were found to have G6PD activity between 20-60% of the normal mean (2.03U/gHb-6.12U/gHb) by OSMMR-D kit and three cases were diagnosed by FST method (Table 5).</div>\r\n<div>\r\n	Racial breakdown showed that 2.28% (14 cases) were Malays, 0.98% (6 cases) were Chinese and 0.16% (one case) were Indian neonates diagnosed by OSMMR-D kit (Table 6). In the female Malay neonates group, only two (0.44%) cases were interpreted as minimal on FST while 12 other cases (2.61%) were normal by this method. All 14 (3.05%) cases of partial G6PD deficiency including two cases diagnosed by FST method were readily detected by OSMMR-D kit. The G6PD enzyme activity levels ranged from 3.17-7.37 U/gHb.</div>\r\n<div>\r\n	Meanwhile, only one (0.81%) case was interpreted as minimal by FST in the Chinese female neonates as compared to all six cases (4.84%) diagnosed by enzyme assay method. The G6PD activity levels for this group range from 4.02-7.10 U/gHb. The female Indian neonate with partial G6PD deficiency was only detected by OSMMR-D kit and not diagnosed by FST method. There were no partial G6PD deficient neonates detected in other ethnic groups.</div>\r\n<div>\r\n	Therefore, the overall frequencies of partial G6PD deficiency detected by FST and OSMMR-D kit were 0.49% and 3.42%, respectively.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Quantitative enzyme assay however is not routinely available in most hospitals in Malaysia. In our Haematology Laboratory, we have had introduced quantitative enzyme assay using OSMMR2000-D kit employing the Hb normalization technique and normal range for G6PD activity for neonates and adults in our laboratory using this kit have been established (Azma et al. 2010). In this study, we established the normal range, the mean and the standard deviations for G6PD activity for paediatric population age 1 month to 12 years old.</div>\r\n<div>\r\n	The overall mean value for paediatric population age 1 month-12 years old was 10.18 &plusmn; 3.36 U/gHb. Comparing the mean normal value of G6PD activity for neonates and adults (12.43 U/gHb and 9.20 U/gHb, respectively) which have been established earlier, (Azma et al. 2010) the mean normal value of G6PD activity in this paediatric population age 1 month-12 years old falls between these two values. This was probably due to presence of circulating young red cells which was known to be higher in neonates and gradually decreased as they get older. The young red cells (reticulocytes) contained higher G6PD activity than mature red cells. Apart from that, contamination with high total white cells during hemolysate preparation in neonates may contribute to higher G6PD activity level in this population.</div>\r\n<div>\r\n	We also compared the mean values for G6PD activity among different age groups in this paediatric population and there was no significant differences (p&gt;0.05). As this study recruited paediatric population age between one month till 12 years old, therefore variation in the haemoglobin level among various groups need to be considered as well. In normal physiology, immediately after birth, the rates of Hb synthesis and red cell production fall fairly steeply to a minimum level by the second month of life, in response to the sudden increase in the tissue oxygenation at birth. In our setting, term babies have mean Hb value of 15g/dL at four weeks and 12.4g/dL by eight weeks and slowly reduced to about 12.1g/dL by 6-8 month of life. However, there is a slight dip in the mean Hb noticed after 6 to 12 month with a mean Hb of 11.6g/dL but later the level sustained between 11 &ndash; 13g/dL. The reticulocytes count fall after birth as erythropoeisis is suppressed and increases to normal values by 6-8 weeks of age. With the rise in 2,3-DPG level and fall in Hb F level in the first few month of life, the haemoglobin-oxygen dissociation curve gradually shifts to the right, which lead to increase in delivery of oxygen to the tissue and hence, ameliorating the effect of falling Hb. Therefore, the mean Hb level in paediatric population between age 1 month and 12 years old varies between 11.6-15.0g/dL. As compared to the neonate population, the Hb and reticulocytes level is higher and therefore the mean for G6PD enzyme level is expected to be higher in this group. This normal physiological fluctuation in Hb levels defines the need for a method that can &lsquo;normalize&rsquo; the Hb level so that the level of G6PD enzyme level will not be affected.</div>\r\n<div>\r\n	The mean normal for G6PD activity between male and female children in various age groups also showed no significant differences (p&gt;0.05). Similar findings also seen in neonates and adult population in a study done by Azma et al. (2010) where it was shown that gender did not affect the G6PD enzyme activity level in their respective population.</div>\r\n<div>\r\n	The comparison of mean normal for G6PD enzyme activity in various ethnic groups showed no significant differences (p&gt;0.05). There were also no significant differences in the mean normal G6PD activity between male and female in various ethnic groups (p&gt;0.05). Similar findings have been described by Azma et al. (2010) in neonates and adult populations, however they only compared two major ethnic groups in Malaysia, namely Malays and Chinese. Ainoon et al. (2003) has also described similar findings by using the Randox G6PD kit.</div>\r\n<div>\r\n	In this study, the cut-off point for G6PD deficiency in paediatric population was 6.12 U/gHb and the range for partial G6PD deficiency was 2.04-6.12 U/gHb. The range for partial G6PD deficiency for neonate and adult population by Azma et al. (2010) were 2.5-7.4 U/gHb and 1.84-5.52 U/gHb, respectively. Therefore the range for partial G6PD deficiency established for the paediatric population from this study was expected to fall in between the neonate and adult ranges.</div>\r\n<div>\r\n	Prevalence of partial G6PD deficiency detected by both FST and G6PD enzyme assays were determined in all 236 subjects. None of the male children (121) had partial G6PD deficient. However there were four male children with severe G6PD deficiency detected by both methods. Their G6PD activity level was &lt;2.04 U/gHb.</div>\r\n<div>\r\n	In the female children group, one out of 115 cases (0.9%) had minimal G6PD activity by FST and was confirmed partial G6PD deficiency by quantitative G6PD enzyme assay. Meanwhile, the quantitative G6PD assay was able to pick up 18 cases (15.7%) with G6PD deficiency including the sample detected by FST. Therefore, the FST misdiagnosed 14.8% subjects as normal. The residual G6PD activity ranged from 41.6%-51.7% (4.23-5.26 U/gHb). These cases were classified as partial G6PD deficiency. A study by Wolf et al. (1987) revealed that sensitivity of FST for detection of partial G6PD deficiency was low (32%) as compared to detection of total male G6PD deficient.</div>\r\n<div>\r\n	In this study, we found that the overall prevalence of G6PD deficiency by FST was 2.12% (5/236), but by using quantitative enzyme assay the prevalence was 9.32%. Therefore, semiquantitative screening test failed to detect children with partial G6PD deficiency but no problems in detecting those with severe G6PD deficiency. These findings were supported by previous study that conducted in neonate population in Hospital Kuala Lumpur which showed that semiquantitative method was able to detect neonate with severe G6PD deficiency if G6PD activity was less than 20% of normal mean (Boo et al. 1994). Therefore, screening with FST was able to diagnose male neonates with severe G6PD deficiency.</div>\r\n<div>\r\n	In this study we also looked at the prevalence of partial G6PD deficiency in 614 female neonates born in UKMMC. The cut-off points for partial G6PD in this population were based on a study done by Azma et al. (2010). The established range for partial G6PD deficiency for this population was 2.5-7.4U/gHb. Subjects with enzyme activity within this range were classified as partial G6PD deficiency. From 614 subjects, 21 (3.42%) had G6PD activity level between 20 - 60% of the normal mean by enzyme assay method and only three cases showed minimal G6PD activity by FST.</div>\r\n<div>\r\n	The prevalence of partial G6PD in female neonates by using FST and quantitative enzyme assay were 0.49% and 3.42% respectively. Therefore, these indicate that 2.93% (18) cases were missed by routine screening test and these cases were found to be partial G6PD deficiency. These findings were supported by a study by Ainoon et al. (2003) involving 976 neonates (both male and female neonates), they found that the overall prevalence by using FST and quantitative enzyme assay were 3.28% and 7.17%, respectively, which conclude that semiquantitative screening method failed to diagnose 3.9% of neonates with G6PD deficiency, and all were female. Another study by Reclos et al. (2000) recruiting 2150 neonates revealed that 24 subjects classified as normal by FST found to be partially deficient by the quantitative enzyme method and they too were all female. The rate of detection was much higher by enzyme method and had good sensitivity and specificity whereby all cases detected by FST were readily diagnosed by this quantitative method. Meanwhile FST tend to give negative result for mild to moderate deficiencies. This could also possibly explain the misdiagnosis of 44% of infants with severe hyperbilirubinaemia in Malaysia (Selvaraju 1999).</div>\r\n<div>\r\n	In conclusion, the OSMMR-D G6PD kit is excellent in detecting partial G6PD deficiency in female population. Since, the FST does not appear to miss G6PD deficient males, it is proposed that for financial reasons, quantitative screening could be limited to female neonates. The test is easy to perform, less laborious, more sensitive, rapid produce results with affordable cost.</div>\r\n', 1),
(1177, 1169, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Urinary incontinence (UI) is &lsquo;the complaint of any involuntary leakage of urine&rsquo; (Abrams 2002). Its prevalence rate in women in the general global population ranges from 20-71 percent, while in pregnant women the rate is 36-65 percent (Burgio et al. 2003; Glazener et al, 2006). Compared to antenatal, postnatal women are susceptible to UI due to various risk factors such as the birthing process itself (Burgio et al. 2003). Although the fertility rate is higher in Eastern countries, including Malaysia, studies on UI in PN women are scarce. In western countries, the prevalence rate in PN women ranges from 11.4-31 per cent (Boyles et al. 2009; Burgio et al. 2003). Only two published studies of PNUI on Malaysian population were found, both conducted with women in the general population, not specific to women of childbearing age (World Health Organization 2003; Zarina et al. 2005).</div>\r\n<div>\r\n	Urinary incontinence (UI) most commonly is a transient condition that would normally resolve within the first three months post-delivery (Burgio et al. 2003; Farrell et al. 2001). In few women, however, if left untreated, the problem persists after delivery resulting in chronic postnatal urinary incontinence (PNUI) (Burgio et al. 2003). The relationship between traditional Malay confinement care (TMCC) practice and postnatal women wellbeing however, had not been previously examined, nor its effect on PNUI. The aim the present study was to investigate the prevalence of postnatal urinary incontinence (PNUI), the risk factors associated with it and postnatal women&rsquo;s (PN) level of engagement in practicing traditional Malay confinement care (TMCC) in Kelantan, Malaysia.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	PARTICIPANTS AND DATA COLLECTION</div>\r\n<div>\r\n	This cross-sectional questionnaire survey was conducted from April to June 2008 in six Maternal and Child Health (MCH) clinics in six districts in Kelantan, Malaysia. Sample size was determined based on the 30% prevalence rates of UI in PN women as reported in the literature (Huebner et al. 2010; Glazener et al. 2006). A one-sample proportion formula for a prevalence study with specified absolute precision was used in this study (Creative Research Systems 2010; Lwanga &amp; Lemeshow 1991). This power calculation, was based on a sampling frame of 420 PN women (available at the time allocated for data collection based on the previous statistical records of each clinic) and allowed for a five percent error, &mu; = 0.05 and 95 percent confidence internal. An additional 10 percent was included to ensure that the sample size was adequately powered to overcome the possibility of drop-outs/withdrawals and missing data (Boriboonhirunsarn 2011). On this basis, it was estimated that 355 PN women were required for the study. Convenience sampling was used to select participants among PN women who attended the clinics for their child immunization during the data collection period. All PN women at three to five months postpartum, aged between 18 to 45 years old and able to read and understand Malay language presenting at the six clinics were eligible for inclusion. Exclusion criteria were :(i) PN women with less than three or more than five months postpartum and (ii) age older than 45 years.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MEASUREMENT TOOLS</div>\r\n<div>\r\n	Prevalence of PNUI</div>\r\n<div>\r\n	The prevalence was assessed through this question &ldquo;Do you ever experience accidentally leakage urine even with a small amount within the last 12 &ndash; 14 months (include before pregnancy)?&rdquo; where participants required to choose the answer i.e. either &lsquo;Yes&rsquo; or &lsquo;No.&rsquo;</div>\r\n<div>\r\n	Associated factors of PNUI</div>\r\n<div>\r\n	Associated factor was assessed within two areas: participants&rsquo; background and their practice of TMCC.</div>\r\n<div>\r\n	Background characteristics</div>\r\n<div>\r\n	Twenty six questions pertained to participants background include maternal age, age at first delivery, education level, and monthly family income. In this section, obstetrics and health histories including smoking and bowel habit, and the type of toilet been used were also asked. Information about the degree of urine leakage during pregnancy and post-delivery was gathered using the Revised Urinary Incontinence ScaleLevel of engagement of Traditional Malay Confinement Care (TMCC)</div>\r\n<div>\r\n	A self-developed scale was used to measure the practice of TMCC among PN women in this study. The questions developed for this scale was based on the 13 TMCC items. Face validity and user friendliness of the scale were confirmed by a group of experts including a gynecologist, three certified midwives and two traditional midwives. Its structural validity and reliability was later tested on the collected data.</div>\r\n<div>\r\n	Written informed consent was obtained before entering this study and no further physical examination and investigation was carried out. A nursing member at each health clinic was assigned to distribute the self-completion questionnaire to eligible participants whilst waiting for their children to be immunized.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	STATISTICAL ANALYSIS</div>\r\n<div>\r\n	Statistical analysis was performed using SPSS 18.0. The prevalence of PNUI and the relationships between PNUI and possible associate factors were analyzed. Data are summarized as means&plusmn; standard deviations or percentages, as appropriate. Chi-square tests were used to analyze categorical variables. Factor analysis was used to identify latent factors underlying the TMCC scale and to test the structure validity of the self-developed scale. Following this, Cronbach&rsquo;s &alpha; test was utilized to assess the internal consistency of items in the TMCC scale. The scores of each factors identified from the TMPC scale between women with PNUI and without PNUI were also compared (body care, nutrition-hydration and rest) using the Mann-Whitney U test in order to identify the differences of engagement in the TMCC in the two groups. As this is a newly developed scale, manual calculation was made to assess the level of TMCC practice by PN women. Scores from all items of the three factors of TMCC scale were range from 0 to 100%, with a higher score indicating higher level of practicing TMPC (0-29% = Not practice; 49 % = slightly practiced; 50-79 % = moderately practiced; 80-100 % = strictly practiced). Potential risk factors for PNUI were identified through a simultaneous logistic regression. A P value of 0.05 was considered as a level of significance.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ETHICAL CONSIDERATION</div>\r\n<div>\r\n	Ethics approval was obtained for the study (Reference: SBRE 3987) from the University Institutional Review Board (Flinders University) and the Government of Malaysia. Permission to conduct the study was also granted by the Medical Research and Ethics Committee, Malaysian Ministry of Health and also from the Director of the six health clinics.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 395 PN women presented in the MCH clinics which fulfilled the inclusion criteria during the study period. Of those, 362 (91.6%) completed the questionnaires and were used as our final analytic sample. The prevalence of PNUI for women who attended six health clinics in Kelantan, Malaysia over the study period was 22.1% (n=80).</div>\r\n<div>\r\n	The majority of PN women in this study (63.3%) were less than 25 years at their first pregnancy (M = 23.1, SD = 4.5). Most participants (40.9%, n= 148) had more than four children (grand multiparous), the average number of children was 2.95 (SD = 1.6) for all participants. Numbers of women in the three postnatal periods were almost equal (three months 31.8%, four months 32.9%, and five months 35.4%). The majority (86.7%) had undergone spontaneous VD for their last delivery, whereas the rest (13.3%) had intervention deliveries (forceps, vacuum and caesarean). The children&rsquo;s birth weight, however, was similar between the two groups, with the average weight of 3.1 (SD = 0.5). The majority (87.3%) were healthy with only 12.7 percent (n=46) reporting they had a medical illness such as hypertension (3.3%, n=12) and diabetes (2.8%, n=10). Consistent with these results, only 31.2 percent said they had taken medication during the study. A contraceptive drug was the main medication taken by the majority of respondents (70.8%, n=80). Only two participants had taken either an antihypertensive drug (0.3%) or a diabetic drug (0.3%). Constipation was rare and the majority of participants (69.1%) used the squatting toilet. Only 15.5 percent (n=56) of the PN women said they had undergone obstetric/gynecological surgery.</div>\r\n<div>\r\n	Of the 13 initial items of TMCC, three main domains were identified through factor analysis: 1) &lsquo;body care&rsquo;, 2) &lsquo;nutrition-hydration&rsquo; and 3) &lsquo;rest&rsquo; as in Table 1. Cronbach&rsquo;s alpha coefficients of the TMCC scale were 0.7, 0.6 and 0.5 for Factor 1, Factor 2 and Factor 3 respectively. The results suggest an acceptable internal consistency of the TMPC instrument with three factors. Mean-inter correlation between 0.2 and 0.4 is viewed as an adequate indicator of internal consistency for a short scale such as the TMCC (Briggs &amp; Cheek 1986; Polit &amp; Beck 2014).</div>\r\n<div>\r\n	TMCC was practiced by all participants (n=362) at some point during their confinement period. The majority (73%, n=266) practiced it moderately. Seventy one (88%) of the 80 women who experienced PNUI said the onset was during pregnancy. Sixty two (76%) of the 80 women who experienced PNUI had stopped PNUI between two days to two months after giving birth, while the remaining 16 women (20%) were still experiencing UI more than three months postpartum during the study period (Figure 1).</div>\r\n<div>\r\n	This revealed that women who had PNUI were more likely to engage in a higher level of &lsquo;Body care&rsquo;. Although vaginal delivery and &lsquo;Body care&rsquo; were indicated as significant in the initial analysis, they were excluded from further analysis due to sampling bias. These includes the difference between numbers of multiparous (70.7%, n=256) and primiparous 29.3% (n=106) in this study. In addition, more PN women had delivered vaginally (86.7%, n=314) and the rest had assisted deliveries (13.2%, n=48). With these limitations, two variables emerged from the study, which could have potential relationship with PNUI (age at first delivery less than 25 years old and monthly family income less than RM1000.00 (USD300)) and other predictor variables identified from the literature were further tested through simultaneous logistic regression to model the risk of PNUI. However, the model was unable to indicate any potential predictor for PNUI, which indicates the needs for further exploration of this issue in future research.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The prevalence rate of PNUI identified in our study is 22.1%. Our results also indicate that 20% of the women in the study who experienced PNUI were still experiencing it a three months post-delivery. This finding is similar to other studies that between 8.2-29% of PN women reported as still experiencing UI at three months post-delivery (Pregazzi et al. 2002; Torrisi et al. 2007; Glazener et al. 2006). Our study also revealed that vaginal delivery has a positive association with PNUI, which is consistent with Ahmadi et al. (2010) and Farrell et al. (2001). In addition, women who had PNUI were more likely to engage in a higher level of &lsquo;Body care&rsquo;, a component of TMPC than those without PNUI.</div>\r\n<div>\r\n	The prevalence rate identified in our study is within the range found in studies in the Western countries. However, our study was unable to clarify causes and types of PNUI due to the use of self-administered survey without clinical assessment. In addition, the definition of UI and the use of a convenience sample may also have impacted on the prevalence rate compared with other studies (Abram et al. 2005; Minichiello et al. 2004). It is acknowledged that determining prevalence rates of PNUI is difficult as there are different definitions, and risk factors of PNUI. Some factors may attributes to these variances such as different sample size and age range of women involved. It is also suggested that while UI in younger women is mostly associated with vaginal delivery, other factors may differs in a much older women. Age and the existence of other diseases may also contribute to the occurrence of UI in this group of women by influencing the healing process and also depending on their body&rsquo;s ability to compensate for the symptom (Glazener et al. 2006).</div>\r\n<div>\r\n	Similar to other studies (Burgio et al. 2003; Farrell et al. 2001), the findings of this study suggests that PNUI is a transient condition for the majority of women who experience it post-delivery. Also, this study have found that a reasonable proportion of the study populations were still experiencing UI at three months post-delivery, as indicated in other studies (Pregazzi et al. 2002; Torrisi et al. 2007). This suggests, much more attention should be paid to this group when planning health care for PN women as they may suffer from persistent UI if the problem remains untreated (Burgio et al. 2003). In order to address clinical management for PN women with persistent UI, it is suggested that a standard time period should be established to determine the prevalence rate for PNUI in future research. Considering there is evidence that indicates the symptom resolves within three months post-delivery and exceeding this period onwards may indicate persistent PNUI, measurement at three and six months postpartum is suggested. The use of a standard period is important to allow comparison between studies on prevalence rates of UI and management outcomes of UI.</div>\r\n<div>\r\n	It is acknowledged that childbearing-age women are susceptible to PNUI because of the many risk factors related to pregnancy and the delivery process. The most common risk factors associated with PNUI includes UI before pregnancy, UI beginning during pregnancy, vaginal delivery, maternal BMI, maternal age of more than 30 years old, and instrumental delivery. Stainton et al. (2005) examined those who were at risk for PNUI following their first pregnancy and birth. The findings showed that urine leakage before the first pregnancy was a key predictor of PNUI. Those with pre-pregnancy UI were 4.14 times more at risk of experiencing PNUI one year after giving birth than those without previous urine leakage (Stainton et al. 2005). Similarly, Fritel et al. (2004) reported that UI before pregnancy was one of the independent risk factors for PNUI [OR18.7; 95% CI 3.6-96.4]. For our study however, only 5% (n=362) of the participants experienced the symptoms before pregnancy.</div>\r\n<div>\r\n	Urinary incontinence that begins during pregnancy is also associated with PNUI. This can be seen in a longitudinal cohort study (Vinktrup et al. 2006) in which the prevalence of postnatal SUI was significantly higher (p&lt;0.01) in those who had UI during the first pregnancy (58%). This finding is supported by other studies where UI during pregnancy was found to strongly predict PNUI (Arrue et al. 2010; Foldspang et al. 2004). However, other research findings showed that the group of women who experienced PNUI was different from the group who experienced UI during pregnancy (Huebner et al. 2010). The findings identified that the women who had PNUI were not the same women who were incontinent before delivery and vice versa in almost 50 percent of cases. PNUI beginning during pregnancy was most commonly reported by PN women in previous studies (Burgio et al. 2003). Similarly, the majority of women who indicated that they experienced PNUI (89%, n=71) in our study said the onset was during pregnancy.</div>\r\n<div>\r\n	It has been reported that vaginal birth damages the tissue of the pelvic floor, mainly affecting the nerve supply to the pelvic floor muscles and therefore their strength. There are three potential causes of injury to pelvic floor muscle tissue; direct muscular injury, direct connective tissue damage, and direct neural damage or damage during remodeling (McLean &amp; Cardozo 2002). Abnormal damage of pelvic floor muscles following vaginal birth may cause morphological and functional changes that are not entirely reversible (Connoly &amp; Thorp 1999) and may later impair the bladder neck support, thereby contributing to the development of PNUI [41]. UI may persist for those who have undergone repeated vaginal delivery (Burgio et al. 2003; Farrell et al. 2001). Vaginal delivery is associated significantly with PNUI (OR 2.360; P=0.002) as reported previously (Burgio et al. 2003). This is similar to our study which showed that delivery method was significantly associated with PNUI (p= 0.03) (Table 2).</div>\r\n<div>\r\n	The components of TMCC share many similarities with traditional care practices in other Asian countries. This includes rest, food, family support and duration of the care practice. The TMCC however, appears to have some unique components such as whole body massage, pelvic area warm compress and warm bath with herbs (Dennis et al. 2007). Our study found that women who had PNUI were more likely to engage in a higher level of &lsquo;Body care&rsquo;, than those without PNUI. These findings reflect that most Malay PN women were obsessed towards TMCC. Since TMCC has been practiced by PN women in Malaysia for so many years now, this finding need to be challenged in future research to confirm its effectiveness to maintain women&rsquo;s overall health including prevention of PNUI. To date, there has been no study published on TMCC and postnatal incontinence. Henceforth, whether TMCC actually has any benefit in PNUI or allow resolution of PNUI earlier is not known.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	IMPLICATIONS FOR PRACTICE</div>\r\n<div>\r\n	There is a requirement for additional professional education programmes in continence-specific care for health professionals (HPs) in the clinical area, particularly those at primary care settings because they would be the first to meet women with urinary symptoms. Accordingly, early assessment of PNUI can be undertaken in the primary healthcare setting because it is a centre of health reference and serves a large number of women in the community. As pre-pregnancy UI and UI during pregnancy are some of the risk factors of PNUI and PNUI itself is a risk factor for long-term UI in women, screening should be done for all women during their first visit to antenatal clinic to identify those at risk for PNUI.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	STUDY LIMITATIONS</div>\r\n<div>\r\n	Due to the nature of descriptive study and sampling bias, this study was unable to explore causes and types of PNUI. In addition, due to small numbers of women with PNUI, future research is suggested with a larger sample size and more in depth study in relation to the prevalence and risk factors of PNUI including the impact of the body care component of PNUI.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In this study, UI is prevalent among PN women in Kelantan, Malaysia. Vaginal delivery was the only variable found to be significantly associated with PNUI. Additionally, the practice of &lsquo;body care&rsquo;, a component of TMCC, was found to have positive association with PNUI.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ACKNOWLEDGMENTS</div>\r\n<div>\r\n	The authors would like to thank all the health professionals in the selected clinics and all the participants of this study; the postnatal women, doctors and nurses. Also to Rukiah, Zaleha and Zarina, Dr. Wan Mohd Zahirudin of University of Science Malaysia and Mr Pawel Skuza of Flinders University for their support and advice. Our appreciation also goes to the Ministry of Higher Education, Malaysia, the University of Science Malaysia and the School of Nursing &amp; Midwifery, Flinders University, South Australia.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1182, 1174, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	It is estimated that about 40,000 people suffer from stroke annually in Malaysia (National Stroke Association Malaysia, 2012). It is the 3rd leading cause of death in the country, losing only to heart disease and cancer. Stroke is a preven disease. The majority of the established risk factors of stroke are preventable or modifiable either through lifestyle modification or medication. Poor knowledge of stroke risk factors and warning signs in the general population has been shown in previous studies. Unfortunately, it has been the poorest in the group with the highest risk of stroke, e.g., those age over 75 (Pancioli et al. 1998; Yoon &amp; Byles 2002; Reeves et al. 2002; Schneider et al. 2003; Carroll et al. 2004; Reeves et al. 2008). When comparing between those who are aware that they have a risk for stroke and those who do not have risk factors, the knowledge of warning signs among them is more or less the same (Sug Yoon et al. 2001).</div>\r\n<div>\r\n	<div>\r\n		Pancioli et al. (1998) reported in their study that only 49% of the respondents named hypertension as a risk factor of stroke, followed by stress (23%), poor eating (20%) and smoking (19%). In term of warning signs, dizziness was identified by 24% of respondents, followed by numbness (19%), headache (16%) and weakness (15%). When specific warning signs based on definition by the National Institute of Neurological Disorders and Stroke were used, 57% of respondents listed at least one of them correctly, 28% listed at least two correctly and only 8% listed three correctly.</div>\r\n	<div>\r\n		The risk profile of stroke differs between men and women. Men are more likely to have cardiac illness and smoking as their risk factors, whereas women are more likely to have hypertension and atrial fibrillation as part of their risk factors. Dearborn et al. in their study of predominantly white, higher income and well educated cohort of 805 women from the Universityof Connecticut Cardiology Center, found that generally these women underestimated their risk of stroke, and the high risk group women did not perceive their risk as being higher that their peers (Dearborn et al. 2009).</div>\r\n	<div>\r\n		Access to emergency services plays a crucial role in determining how fast a stroke sufferer can reach the hospital. Between 27-100% of respondents reported in several studies that they would call the emergency medical services if they developed symptoms of stroke (Sug Yoon et al. 2001; Reeves et al. 2002; Cheung et al. 1999). However, in a study among stroke sufferers, it was noted that only 18% had actually called the emergency medical services (Sug Yoon et al. 2001). A study performed among members of the public in major cities in Brazil reported that the knowledge of the emergency telephone number was alarmingly low (Pontes-Neto et al. 2008). It was reported that only 34.6% of the 814 respondents knew about the nationwide emergency number. In Malaysia no such studies have been performed. Another obstacle to early treatment is the emergency response time. In Malaysia the reported emergency response time is 21.1 minutes, compared to seven minutes in the United Kingdom and 11 minutes in New York (Shah et al. 2008). Another factor that may affect the time to treatment is the living condition. Few of the stroke sufferers at the time of onset of symptoms are alone at home, hindering them from seeking treatment immediately.</div>\r\n	<div>\r\n		Public education campaigns has brought to significant improvement in awareness of stroke warning signs among the population (Schneider et al. 2003; Reeves et al. 2008; Fogle et al. 2008). However, this improvement was seen less effective in older people (Reeves et al. 2008; Silver et al. 2003). Silver et al. (2003) in their study looking at advertising strategies to increase public awareness on stroke found that the most effective media was television. People who were exposed to television campaigns were able to name more than two warning signs of stroke. It was also noted to be effective in increasing knowledge among men regardless of their high school education level. However, it was found not to be effective among the senior population, the group with highest risk of stroke. Stroke screening and education programmes are also important tools in increasing awareness. In a study done by Stern et al, the ability of participants to recognise stroke symptoms, namely weakness, numbness on one side, difficulty talking or understanding speech, and difficulty with vision, improved from 59% to 94% immediately following intervention but then declined to 77% when tested again three months later (Stern et al. 1999). Silver et al. (2003) similarly found that intermittent low level advertising is more effective than the intensive one off approach. This study aimed at addressing the issues of stroke awareness among the public and to ascertain our achievement in stroke prevention.</div>\r\n	<div>\r\n		MATERIAL AND METHODS</div>\r\n	<div>\r\n		GENERAL OBJECTIVE</div>\r\n	<div>\r\n		To assess the public&rsquo;s knowledge of risk factors, symptoms, treatments and source of information in relation to stroke and transient ischaemic attack.</div>\r\n	<div>\r\n		SPECIFIC OBJECTIVE</div>\r\n	<div>\r\n		1. To determine the level of knowledge on stroke or transient ischaemic attack (TIA) warning signs, symptoms, treatments and source of information among the community dwellers.</div>\r\n	<div>\r\n		2. To characterise stroke risk profiles among the respondent and correlate them to their knowledge of stroke warning signs and risk factors.</div>\r\n	<div>\r\n		STUDY POPULATION</div>\r\n	<div>\r\n		All adults attending the two community- based programmes: Community Stroke Awareness Day and Community Open Day, gave their consent to be included for the study.</div>\r\n	<div>\r\n		STUDY SITE</div>\r\n	<div>\r\n		1. Community Stroke Awareness Day, Primary Health Clinic site.</div>\r\n	<div>\r\n		2. Community Open Day, Taman Saujana Impian.</div>\r\n	<div>\r\n		STUDY DURATION</div>\r\n	<div>\r\n		November 2013- January 2014</div>\r\n	<div>\r\n		INCLUSION CRITERIA</div>\r\n	<div>\r\n		Individuals above 18 years of age</div>\r\n	<div>\r\n		EXCLUSION CRITERIA</div>\r\n	<div>\r\n		1. Individuals who were not willing to participate (not consented)</div>\r\n	<div>\r\n		2. Individuals who were mentally challenged</div>\r\n	<div>\r\n		3. Individuals who did not comprehend English or Bahasa Malaysia.</div>\r\n	<div>\r\n		STUDY DESIGN</div>\r\n	<div>\r\n		This was a cross sectional questionnaire based survey. The study questionnaire was designed by an expert panel consisting of Family Medicine Specialists, Emergency Medicine Specialist and a Clinical Epidemiologist based on comprehensive literature review, theoretical concepts and expert knowledge. A modified Delphi process was used to achieve a consensus about the relevance of each question that was included in the questionnaire. The questionnaire was pre-tested on 10 individuals for face validity: to test the flow and comprehensibility of the questions, and time required to complete it. Queries and feedback from respondents were incorporated into the revision of the questionnaire. The finalized questionnaire was then administered to the participants for data collection.</div>\r\n	<div>\r\n		During the programmes, participants were approached during the registration and were invited to participate in the study. Those who agreed were given further information by investigators prior to signing the consent form. Each participant was then given the questionnaire the first two parts of which has to be filled up before presenting to the screening station.</div>\r\n	<div>\r\n		At the screening station, the staff in charge completed the questionnaire by recording the relevant investigation results i.e. the blood pressure measurement, heart rate recording, physical measurement to determine BMI and blood sugar level in the form.</div>\r\n	<div>\r\n		Completed questionnaires were then collected and respondents were given a copy of the screening information that contained their investigation results and their stroke risk with explanation of the information by the staff. Those who were at high risk of stroke were referred for further investigation and follow up at the Primary Health Clinic Centre.</div>\r\n	<div>\r\n		STUDY TOOL</div>\r\n	<div>\r\n		There were three parts to the questionnaire. The first part of the questionnaire gathered demographic information and personal medical history.</div>\r\n	<div>\r\n		The second part of the questionnaire assessed knowledge of stroke warning signs or symptoms, risk factors for stroke, emergency telephone number in Malaysia, treatment for stroke and source of information.</div>\r\n	<div>\r\n		The third part of the questionnaire assessed the respondents risk for stroke or TIA through health screening using the National Stroke Association checklist (Figure 1) and relevant investigations (blood pressure measurement, heart rate recording, physical measurements to determine BMI and blood glucose level) by the staff nurse. Each factor carried one point within a particular category (high risk, caution or low risk), depending on the level measured or presence/absence of the factor. The cumulative score of each category determined whether the participant had high or low risk for stroke (Figure2).</div>\r\n	<div>\r\n		ETHICS APPROVAL</div>\r\n	<div>\r\n		Ethics approval was obtained from the Universiti Kebangsaan Malaysia Medical Centre (UKMMC) Research Ethics Committee.</div>\r\n	<div>\r\n		STATISTICAL ANALYSIS</div>\r\n	<div>\r\n		All data were entered into SPSS version 20 for analysis. Descriptive analysis was done on the demographic data and presented as mean with standard deviation or median with inter quartile range depending on its normality distribution. The categorical data were expressed as frequency and percentages. Score for knowledge was presented as mean (standard deviation) or median (inter quartile range) and the cut off score was determined based on the upper 25th centile. Categorisation of the Stroke Risk Score was presented in frequency and percentages based on the category as provided by the American Stroke Association (National Stroke Association, 2014). The Chi Square test was used to assess the association between knowledge of stroke symptoms and risk factors and the patient&rsquo;s own risk of stroke based on the Stroke Risk Card.</div>\r\n	<div>\r\n		RESULTS</div>\r\n	<div>\r\n		A total of 112 questionnaires were distributed during the two day events. Out of this 12 were excluded &ndash; 10 were incomplete while the other two were excluded because the respondents were below 18 years of age.</div>\r\n	<div>\r\n		DEMOGRAPHICS</div>\r\n	<div>\r\n		All of the 100 respondents were Malaysians. The age of the respondents ranged from 20-82 years, with a mean age of 50.88 years. Forty per cent of the respondents were men with majority being Malay (93%), followed by Chinese (3%), Indian (2%) and other races (2%).</div>\r\n	<div>\r\n		The majority of them had some form of formal education with 12% having basic education. Twenty two percent of respondents were unemployed and in terms of social support, 62% were married. Table 1 showed the baseline characteristic of the respondent.</div>\r\n	<div>\r\n		MEDICAL HISTORY</div>\r\n	<div>\r\n		Forty seven percent of respondents reported that they had a medical illness. The three commonest medical conditions reported were hypertension (33%), hypercholesterolaemia (24%) and diabetes mellitus (14%). No respondent suffered from any cardiac rhythm disturbances, such as atrial fibrillation. It is possible that some of them were not aware of their irregular heart rhythm or did not understand about the condition.</div>\r\n	<div>\r\n		Overall, the mean systolic and diastolic BP measured were within the acceptable range with the mean SBP being of 128.5mmHg and median DBP 77mmHg. The median blood glucose level was 6.25mmol/L. Only one of the respondents fasted. The majority of the respondents did not perform regular exercise. Only 25% reported that they exercised regularly. The median for BMI was 26. Most of the respondents did not smoke (91%) at the time of the study &ndash; 1 respondent had quit smoking. Table 2 detailed the clinical characteristics of the respondents.</div>\r\n	<div>\r\n		KNOWLEDGE OF STROKE</div>\r\n	<div>\r\n		Warning Signs and Management</div>\r\n	<div>\r\n		Quite a high number of respondents were able to identify at least one warning sign of stroke. The commonest identified warning sign was altered sensation of one limb (57%), followed by facial asymmetry (41%) and arm weakness (32%). More than a quarter of the respondents (27%) were unable to identify even a single warning sign. We included a few distracters in the list of warning signs. Most of the respondents were able to exclude them. Chest pain and dyspnoea were the top two that were commonly mistaken for stroke symptoms in 29% and 22% of respondents respectively. The commonest risk factors identified by respondents were hypertension (68%),followed by lack of exercise (45%) and alcohol use (43%). The non-modifiable variables like menopause (5%) and male gender (9%) were least likely to be identified as risk factors.</div>\r\n	<div>\r\n		Figure 3 showed the distribution of the risk factors. Seventy eight percent of respondents were able to correctly identify the EMS number. Only 11% of the respondents said that they are aware of the emergency treatment of stroke but when asked to elaborate, no one was able to do so. More than half (58%) thought rehabilitation was an important part of stroke management. When asked whether stroke sufferers can have full recovery, 53% said &lsquo;yes&rsquo; but 42% did not know. The majority of the respondents reported that their source of information was from doctors &ndash; hospital based and GP. Among the mass media options, television was the most popular source with 43% reported it as their source of information. Figure 4 represented the distribution of the source of information used by the respondents.</div>\r\n	<div>\r\n		STROKE AWARENESS</div>\r\n	<div>\r\n		In assessing their awareness of the warning signs of stroke, we classified those who were only able to identify less than three signs as having unsatisfactory awareness and those who were able to identify at least three signs to have satisfactory awareness. Only 35% of respondents had satisfactory awareness of stroke. When we analysed according to the risk group, the percentage of respondents with satisfactory awareness in the low, caution and high risk group were 34%, 40% and 35% respectively.</div>\r\n	<div>\r\n		Awareness about the risk factors contributing to stroke was also low. We classified those who were able to identify at least five modifiable risk factors as having satisfactory awareness. Those who identified less than five modifiable risk factors was classified as unsatisfactory awareness. A total of 29% of the respondents were deemed to have satisfactory awareness of the risk factors. Analysis within the risk groups showed a 26%, 30% and 41% satisfactory awareness in the low, caution and high risk groups, respectively. However, this was not statistically significant (p=0.46) (Table 3).</div>\r\n	<div>\r\n		DISCUSSION</div>\r\n	<div>\r\n		This study was carried out at two sites within the Cheras locality-UKMMC Primary Health clinic site and Taman Saujana Impian, on two separate days. Stroke awareness among the respondents was noted to be low both in terms of risk factors and warning signs. This is true across the three risk groups. This finding is not surprising and consistent with other studies (Pancioli et al. 1998; Yoon &amp; Byles 2002; Reeves et al. 2002; Schneider et al. 2003; Carroll et al. 2004; Reeves et al. 2008). In the study by Pancioli et al. (1998) hypertension was commonly identified as a risk factor for stroke followed by stress and poor eating. However, in this study commonly identified risk factors were hypertension, followed by hypercholesterolaemia and diabetes mellitus. This is rather encouraging in a sense that this study population may have some insight into these chronic illnesses and their complications. The most commonly identified warning signs or early symptoms of stroke were altered sensation of limb, facial asymmetry and limb weakness. It is worrying to note that stroke awareness is low and more worrying that more than a quarter of the respondents were unable to identify a single risk factor and warning sign from the given list.</div>\r\n	<div>\r\n		The respondents&rsquo; knowledge on the EMS was encouraging as the majority of them knew the access number. This is in contrast to the study done in Brazil (Pontes-Neto et al. 2008) where such important knowledge was lacking. We should continue to educate the public that early access to EMS is crucial. However, when it came to treatment of stroke, the feedback was somewhat mixed. We concluded that all of our respondents were unaware of the emergency treatment of stroke and its availability. Furthermore, only about half of the respondents were aware that stroke sufferers could make full recovery, while the others were not sure. Similarly, about half felt rehabilitation was a key factor for recovery. All these factors can certainly hamper any effort directed at improving outcome of patients with stroke. These findings highlight the fact that the problem is not just lack of information, but also misperception. This lack of knowledge and misconception need to be addressed and rectified if we want to improve our stroke outcome.</div>\r\n	<div>\r\n		Most of the respondents reported that their main source of information was medical professionals-form direct contact as well as health talks. This highlights the important role we, the healthcare providers, have on educating the public. Sadly, with the increase in patient numbers, healthcare providers are spending less time with, and explaining less to the patients. Among the media sources, television was the most common source used by respondents. This is consistent with the study done by Silver et al. (2003).</div>\r\n	<div>\r\n		There are a few limitation of this study. Firstly, the sample size of the study was small to be able to deduce any statistically significant findings. We should allocate more time and choose more sites that have a better mix of respondent ethnicity to be able to obtain a larger sample size representative of the general population.</div>\r\n	<div>\r\n		Secondly, as we could not conduct bedside tests for blood cholesterol level, we made assumption that those who reported that they had hypercholesterolaemia, would have high blood cholesterol levels. Bedside electrocardiography (ECG) was not available and feasible at a medical screening station; hence arrthymias such as paroxysmal atrial fibrillation can be missed.</div>\r\n	<div>\r\n		CONCLUSION</div>\r\n	<div>\r\n		In conclusion, stroke awareness among our study population was alarmingly low. A more proactive approach to educating the public is needed in order to improve the outcome of stroke sufferers. Health campaigns regarding stroke risk factors, early signs/symptoms and management (emergency and long term) need to be run more regularly targeting the public generally, and moderate and high risk groups specifically. The public need to know about the availability of emergency treatment of stroke that leads to better outcome for stroke sufferers. Healthcare providers need to realise the important role they have in educating their patients as well as the public. In order to do so, they themselves have to be updated on the issue at hand. Television advertisements are good and effective means of education.</div>\r\n</div>\r\n', 1),
(1194, 1186, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Thalassaemia and haemoglobinopathies not only result in serious health problem, but also creates a heavy economic burden to the country (George 2001). Therefore, many countries including Malaysia have established thalassaemia screening programmes (HKCOG 2003; Samavat &amp; Modell et al. 2004; MOH 2009) in order to identify carriers. These programmes mainly focussed on couples of childbearing age either before or during pregnancy. It was also offered as a premarital screening in order to help carriers to make informed decision on the choice of marital partner, since marriage between carriers is not encouraged. Since 2004, Malaysia has carried out screening activities through two different approaches on volunteer participants. In cascade screening, theclose relatives of the patients would be tested, while in target screening, it may involve expecting mothers, adolescents in schools and colleges, and also those who are attending the government health care centres (MOH 2009). However, even though the screening for thalassaemia is free of charge, the response rate is poor in our nation due to lack of awareness, especially in lower educated individuals (Anjanna et al. 2011; Wong et al. 2011).</div>\r\n<div>\r\n	While campaigns are consistently carried out to improve public awareness and to encourage target groups to participate in voluntary thalassaemia screening, an alternative and a feasible approach would be to screen blood donors. In our practice, the blood donors are clinically assessed for medical illnesses through standard questionnaires and simple physical examinations. The pre-donation haemoglobin level is also measured with minimal cut-off of 12.5 g/dL as the acceptance criteria. Prospective donors who are healthy with haemoglobin levels within the normal range are permitted for blood donation. In order to prevent the adverse effect of blood donation to the donors as well as the recipients, the donated blood is always screened for transfusion-transmissable infections. Regarding these sequences of donor selection and screening tests, we might miss donors with thalassaemia trait because carriers of thalassaemia are asymptomatic in general and their haemoglobin values are sometimes above the minimal requirement (Bryant et al. 2009). In the present study, thalassaemia screening was carried out on post-donated blood to find out the prevalence of thalassaemia carrier among healthy blood donors.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	A cross-sectional study was conducted on routine diagnostic samples sent for thalassaemia sceening in Institute for Medical Research. A total of 738 samples from Malaysian blood donors were received from Tengku Ampuan Rahimah Hospital from July until September 2010. The donors were explained about the purpose of the study and consent were obtained from each of them prior to the blood taking. They were selected according to the standard criteria prepared by the hospital including age between 18 and 65 years old, minimum weight of 45 kg and minimum of five hours sleep. Healthy donors with acceptable haemoglobin level were allowed to donate when the time interval from the last donation was at least eight weeks. The acceptable haemoglobin level was presumed ranging from 12.5 to 18.0 g/dl if the donors passed the copper sulphate gravimetric test.</div>\r\n<div>\r\n	Five ml of venous blood specimen were collected in ethylenediaminetetraacetic acid (EDTA) tube during the blood donation process. Full blood count was performed by an automated haematology analyzer, Sysmex XE-2100D and Sysmex XE-2100 Alpha (Sysmex, Japan) in the hospital on the day of collection, while further analyses were conducted in Institute for Medical Research on the next day (within 48 hours). We used Bio&ndash;Rad VARIANT II Haemoglobin Testing System (Hercules, USA), a fully automated cation-exchange high performance liquid chromatography (HPLC) for haemoglobin analysis. Agarose gel electrophoresis using Sebia Hydrasis (Sebia, France) at alkaline pH (8.6) was only done on samples with detected haemoglobin (Hb) variants on HPLC.</div>\r\n<div>\r\n	The cut-off level for haemoglobin A2 of more than 4.0% was used for presumptive diagnosis of classical &beta;-thalassaemia trait (George et al. 2001). But if it was more than 10%, a diagnosis of haemoglobin Lepore was considered, while the level more than 25% was interpreted as suggestive of haemoglobin E trait (Barbara 2006; Ryan et al. 2010). However, when the level was between 16 to 25%, haemoglobin E with alpha thalassaemia, co-inheritance was suggested (Barbara 2006; Ryan et al. 2010). The presence of other abnormal peaks in HPLC chromatogram was correlated with the demographic data and gel electrophoresis result.</div>\r\n<div>\r\n	All statistical analyses were conducted using the Statistical Package for Social Sciences for Windows SPSS 17.5 (SPSS inc., Chicago, IL, USA). The results were expressed as mean + 2 standard deviation (SD).</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Among 738 blood donors, 606 (82%) were males and 132 (18%) were females. The mean age was 33 years and SD was 10. The youngest donors were aged 17 years while the oldest were 61 years. Four hundred and thirteen (56%) were Malays, 162 (22%) were Indians, 148 (20%) were Chinese while 15 (2%) were others. The other races consisted of Iban 2, (0.3%), Kadazan 1 (0.2%), Kayan 1 (0.2%), Bugis 1 (0.2%) and Malaysian-Indonesian 1 (0.2%); while 9 (1.2%) donors did not specify their ethnicity. Seventy four (10%) of the blood donors were confirmed to have haemoglobin levels below 12.5 g/dL (8.7-12.4 g/dL), where four (5.4%) had haemoglobinopathies. Mean corpuscular haemoglobin (MCH) level lower than 27 pg was observed in 225 (30.5%) donors, of whom 35 (15.6%) displayed abnormal haemoglobin fractions on HPLC. Interestingly, haemoglobinopathy (haemoglobin A2 prime) was found in one (0.1%) donor whose MCH value was within the normal range (28.2 pg) but the mean corpuscular volume (MCV)value was 79.7 fL. The other types of haemoglobinopathies detected with their haematological parameters shown in Table 1. All the abnormal cases were Malays except for the &beta;-thalassaemia trait cases, three (60%) were Indians and two (40%) were Malays.</div>\r\n<div>\r\n	&alpha;&alpha;-thalassaemia and normal haemoglobin A2 &beta;-thalassaemia carriers were not be able to be excluded in 190 (25.7%) cases based on our screening tests. The diagnosis would only be confirmed by molecular analysis. Male donors were predominant in this group 140, (73.7%). Malays accounted for 104 (54.7%), Indians 65 (34.2%) Chinese 20 (10.5%) and others one (0.5%). Fourty six (24.2%) donors had haemoglobin less than 12.5 g/dL (8.7-12.4 g/dL) and the median haemoglobin level was 11.6 g/dL. The haematological parameters for these 190 undiagnosed cases shown in Table 2.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The severe types of thalassaemia that pose major public health problem in Malaysia are homozygous &alpha;&alpha;0-thalassaemia (haemoglobin Bart&rsquo;s hydrops foetalis) and &beta;-thalassaemia major (Ainoon &amp; Cheong 1994). Homozygous &alpha;&alpha;0-thalassaemia is associated with perinatal mortality and potentially causing fatal pregnancy complications in mothers. Children with &beta;-thalassaemia major are always manifested with severe anaemia after one year of age requiring lifelong blood transfusion. Since, they are transfusion dependent, iron chelators are important to overcome iron overload</div>\r\n<div>\r\n	complications. However, prior to year 2005, less than 20% of them receive adequate treatment (George 2001). This not only affects the clinical health status, but also gives a great impact on our socio-economy. Hence, establishment of screening programme for identification of thalassaemia carrier is extremely crucial. The most effective approach is the implementation of screening programme among school students, premarital couples, family members of thalassaemia patients and pregnant women during antenatal booking (Bianco et al. 1984; Samavat &amp; Modell 2004; AlHamdan et al. 2007). But a feasible option is to also test blood donors as the blood sample is readily available at time of blood donation. In general, this approach can enhance our national screening programme since the response rate is poor in our community (Anjanna et al. 2011; Wong et al. 2011). Lack of knowledge and unwillingness for screening were the main reason for the poor response (Anjanna et al. 2011; Wong et al. 2011).</div>\r\n<div>\r\n	Few studies reported the success of thalassaemia screening programme at school but few have raised arguments against this practice (Troitskaia et al. 1999; AlArrayed et al. 2003; Li et al. 2009). School students are considered legally minors. Their assents in participating carrier testing may be influenced by friends, teachers and guardians (Ross 2006). The possibilities of confusion between carrier state and being affected by the disease might occur in the absence of proper counselling and this can lead to adverse psychological reaction in those with positive results (Axworthy et al. 1996; Marteau et al. 1992; Stewart-Brown &amp; Farmer 1997). Based on ethical issues, genetic testing like thalassaemia screening can only be carried out if immediate diagnosis is necessary, for instance when an adolescent is pregnant or planning for pregnancy (ASHG/ACMG 1995). At this age, screening would be acceptable. The test should be deferred until they are able to justify the appropriate time to have the screening (Frumkin &amp; Zlotogora 2008). Adolescents aged 18 and above are generally legal for consent because (ASHG/ACMG 1995) they are considered mature and capable in making judgment for their future. Married blood donors who are identified as thalassaemia trait would alert their spouses to get a similar test in order to ascertain their carrier status. If they are at risk of having children with thalassaemia major, they would then be counselled to undergo prenatal diagnostic test, so that they can have the option of either continuing or terminating the affected pregnancy. This, however results in difficult decision making caused by misapprehension of the disorders, cultural values and religious belief (El-Hazmi 2009; Wong et al. 2011). Premarital screening would be an alternative way since this approach might be more effective in the Malay community. It may identify carrier couples, thus giving them the opportunity to circumvent affected pregnancies. Studies showed that the incidence of infants born with the disease reduced tremendously by implementation of premarital screening (Zlotogora 2009).</div>\r\n<div>\r\n	In the present study we found that 36 (5%) of 738 healthy blood donors were presumptively diagnosed as thalassaemia carriers. The frequency of haemoglobin E carrier was similar with the previous study (George &amp; Khuziah 1984), but the distribution of &beta;-thalassaemia carrier and heterozygous haemoglobin Constant Spring were below the reported incidence (George 2001; Wee et al. 2009). &alpha;&alpha;-thalassaemia and normal haemoglobin A2 &beta;-thalassaemia could not be excluded in 190 (25.7%) blood donors in this study because no abnormal pattern was noted in HPLC result. Further confirmation by DNA analysis on these cases demand for futher analysis in the future.</div>\r\n<div>\r\n	Anaemic blood donors are no longer an issue, because further workup will be initiated to find out the cause of anaemia. Nonetheless, for those who pass copper sulphate test, they are always considered to have normal haemoglobin level. Therefore they will be permitted for blood donation and missed from detection of thalassaemia trait. A few studies had found this method was less sensitive in detecting anaemia compared with other portable haemoglobinometry devices available in the market (Newman 1997; Nadarajan &amp; Eow 2002; Boulton 2008). It is not cost-effective to include haemoglobin analysis as routine blood test in blood donors in order to screen for thalassaemia carrier. Besides, it is troublesome to potential blood donors and can negatively impact the blood donor return. In this study, majority (70%) of the blood donors had results within normal limits. A local study which included anaemic blood donors also found that 84% of them had normal haemoglobin electrophoresis pattern (Rosline et al. 2006). Therefore, performing haemogram on initial diverted blood from collateral pouch after blood donation would be more worthwhile. More accurate haemoglobin level would be measured and more detail blood cell indices would be scrutinized. Based on these results, certain donors will be selected for complete haemoglobin analysis. According to British Committee for Standards in Haematology (British Journal of Haematology 2010), MCH value is the recommended parameter for thalassaemia screening as it is more stable than MCV. We had only one case with detected haemoglobin variant on HPLC, yet the MCH value was normal (more than 27 pg) while the MCV value was 79.7 fL. A previous study that adopted MCV level of less than 80 fL as a criterion for further analysis in apheresis blood donors had found that 36% of the donors had haemoglobinopathy (Bryant et al. 2009). Such findings showed that both MCV and MCH values are essential for prediction of thalassaemia trait.</div>\r\n<div>\r\n	It is highly debated whether or not thalassaemia and haemoglobinopathy screening should be done for regular blood donors to avoid transfusion of dysfunctional red cells and transfusion acquired haemoglobinopathy. Rational for screening most often depends on the demographic frequency of thalassaemia and clinically significant haemoglobinopathies in a given population and special requirement of recipients. In addition to this, post transfusion lifespan of erythrocyte is also an important factor in transfusion medicine. Erythrokinetic of thalassaemic red cells in a normal recipient showed 25 to 50% of transfused cells were destroyed within 10 to 20 days rendering the transfusion relatively ineffective (Kaplan &amp; Zuelzer 1950). Similarly, haemoglobin E, an unstable haemoglobin with increased sensitivity to oxidants showed a shortened red cell survival. In United Kingdom, testing of blood cell products for haemoglobin S are undertaken when the prospective recipients are in paediatric age group and neonatal patients (JPAC 2005). However, this policy is deferred at the Northern Ireland Blood Transfusion Services until contemporary ethnicity was fully understood (Northern Ireland Blood Transfusion Service, 2012). A local study may be necessary to assess the effect of transfusing thalassaemic blood in patients before considering such screening test in our blood products.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Thalassaemia screening among healthy blood donors could benefit the existing thalassaemia screening programme in Malaysia. It is a feasible and effective approach in detecting thalassaemia carriers in our population. However, a more cost-effective screening method is suggested since prevalence of thalassaemia carrier in healthy blood donors are low. Haemogram is an appropriate screening test as scrutinisation of blood indices may allow selection of blood donors for further analysis of haemoglobin.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	This work was supported by an operational budget from Haematology Unit, Cancer Research Centre, Institute for Medical Research. The authors wish to thank the Director General of Health, Malaysia for permission to publish this specific paper. We also like to thank the Deputy Director General (Research &amp; Technical Support, MOH) and the Director of IMR for their support. The authors would like to declare that there was no conflict of interest in this study.</div>\r\n', 1),
(1200, 1192, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Throughout the world, cancer involving the female genital tract is a significant cause of morbidity and mortality worldwide. In the United States of America, ovarian cancer is one of the most deadliest and common gynecological cancer in female. In Malaysia, the most common gynaecological cancer is cervical cancer, followed by ovarian cancer. According to The National Cancer Registry 2003 for Peninsular Malaysia showed that over 1,500 women developed cervical cancer of which 700 died each year. Yet, cervical cancer is the most preventable cancer in the general population. According to the National Cancer Registry, ovarian cancer happens to be the fourth most common cancer amongst females in Peninsular Malaysia and accounts for 5% of all cancer cases seen in female (Lim &amp; Halimah 2004).</div>\r\n<div>\r\n	There is high prevalence of gynaecological cancer in Malaysia. However, proper research on the sexual dysfunction affected by the cancer is still lacking. In community-based studies, female sexual dysfunction is found to be highly prevalent in many countries and it ranges between 25% - 63% (Laumann et al. 1999; Brock et al. 2003). A prevalence of 30% has been reported in many Asian countries such as Hong Kong, Japan, Korea, Taiwan and Singapore (Nicolosi et al. 2005). Female sexual dysfunction can be categorized into sexual desire disorder, sexual arousal disorder, sexual lubrication problem, sexual orgasmic disorder, sexual satisfaction problem and sexual pain disorder (American Psychiatric Association 1994).</div>\r\n<div>\r\n	There are reports of sexual complaints in up to 90% of females diagnosed with cancer (Anderson 1990). With the cancer affecting the female genitalia organs directly, it is believed that its prevalence among gynaecological cancer patients is even higher, ranging from 40 to 100% (Casey 1996). The cancer effects such as vaginal dryness, pain or discomfort during penetration and smelly vaginal discharge can cause emotional distress (Taylor 2000). Dysfunction may also result from the treatment that is aimed at curing or containing disease such as radiotherapy and chemotherapy, or as a result of the psychological distress experienced by the patient and/or her partner during diagnosis and treatment (Amsterdam &amp; Krychman 2008; Schover 1997; Schover &amp; Jensen 1998; Anderson 1999). Other than gynaecological cancers, pain, medication, antidepressants, medical conditions such as diabetes, stroke, neurological problem and pain of osteoarthritis can cause sexual dysfunction direct or indirectly. To highlight, the causes of sexual dysfunction in gynaecological cancer patients are multidimensional and all of the possibilities for multiple causations or risk factors need to be reviewed.</div>\r\n<div>\r\n	Majority of gynaecological cancer patients with sexual dysfunction were untreated due to many reasons (Schover 1997). In the busy clinic and ward setting, health care providers and patients may not have the luxury of time and privacy to discuss about their concerns (Marwick 1999). Doctors and patients may also have their own personal discomfort to discuss sexual matters. This can be more obvious when the physician is a male doctor. Patients&rsquo; husbands are also excluded most of the time during assessment of sexual function. The doctors&rsquo; interviews and assessments may be limited by their lack of expertise and knowledge.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Following approval by the Department of Psychiatry, ethics committee of Hospital Universiti Kebangsaan Malaysia (HUKM) and National Medical Research register (NMRR) necessary permission was obtained from the Head of Department of Gynae-oncology Hospital Sultanah Bahiyah, Alor Star, Malaysia. The samples were eventually gathered over a period of five months. The sample comprised patients diagnosed with gynaecological cancer who were receiving treatment. Eligibility criteria included age 18 years or older, ability to communicate effectively and to give informed consent. Exclusion criteria included patients with history of drug abuse, significant cognitive impairment, patients who refused to give consent and unable to communicate. Subjects were interviewed using questionnaires that included socio-demographic variable (i.e. age, ethnicity, religion, marital status, occupation, education level, husband&rsquo;s help in the household, presence of husband and children less than 18 years old at home and perceived social support) and medical variable (i.e. type of cancer, staging, cancer treatment modalities, pain perception, perceived social support, medical illness and psychiatric illness). Furthermore, three more questionnaires such as Mini International Neuropsychiatry Interview (MINI), Malay Version Sexual Female Sexual Function Index (MVFSFI) and World Health Organization Quality of Life (WHOQOL-BREF) were used. MINI was developed by Sheehan et al. (1998), MVFSFI and Malay version of WHOQOL-BREF have been validated and used previously in other studies (Sidi et al. 2007; Hasanah et al. 2003). In MVFSFI, the cut off total score for sexual dysfunction was &le;55 and the cut off score for each domain of sexual dysfunction: &le;five for the sexual desire disorder (sensitivity 95% and specificity 89%); nine for sexual arousal disorder (sensitivity 77% and specificity 95%); 10 for disorder of lubrication (sensitivity 79% and specificity 87%); four for orgasmic disorder (sensitivity 83% and specificity 85%); 11 for sexual dissatisfaction (sensitivity 83% and specificity 85%); and &le; seven for sexual pain disorder (sensitivity 86% and specificity 95%). All data were analysed using the Statistical Package for Social Sciences (SPSS) version 11.5 computer program.</div>\r\n<div>\r\n	Initially the individual effects of the socio-demographic and medical variables were tested. Student t-Test was used to analyze normally distributed quantitative variables such as age group. To analyze not normally distributed quantitative variables such as cancer duration, duration of absence of sexual intercourse among married patients, scoring of the MVFSFI and WHOQOL-BREF, Mann-Whitney U test was performed. Binary logistic regression test was used to analyze association between sexual dysfunction and those variables that deemed significant in the univariate and bivariate tests. Socio-demographic and medical variables that deemed significant (such as education level, duration of cancer, ongoing chemotherapy, pain perception and quality of life aspects such as physical health, psychological health and social relationship) were concurrently entered into the logistic regression.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Eighty-three out of 120 (69%) patients were married. The rest (37, 31%) were single, widowed, separated or divorced. All patients from the latter group denied having sexual partners and were excluded in the assessment of sexual dysfunction. With the cut-off point 55 or less for sexual dysfunction, the prevalence of sexual dysfunction was 65% (54/83).</div>\r\n<div>\r\n	The number of patients with desire dysfunction was 64 (77%), sexual arousal dysfunction (54, 65%), sexual lubrication problem (59, 71%), orgasmic dysfunction (47, 57%), sexual satisfaction problem (58, 70%) and sexual pain problem (47, 57%).</div>\r\n<div>\r\n	With regards to the cancer subjects who were married, 44 out of 83 (53%) married patients did not have sexual intercourse with their spouses for more than one month. The duration of absence of sexual intercourse ranged from 2 months to 240 months (median = 12 months) (IQR = 5 to 24 months).</div>\r\n<div>\r\n	Table 1 and 2 represents the relationship of sexual dysfunction with psychosocial variables, medical variables and domains of quality of life.</div>\r\n<div>\r\n	It depicts that sexual dysfunction was significantly associated with education level (OR 3.055, 95% CI 1.009-9.250), duration of cancer (OR 0.966, 95% CI 0.966-0.998), ongoing chemotherapy (OR 3.045, 95% CI 1.149-8.067), pain perception (OR 3.230, 95% CI 1.257-8.303), absence of sexual intercourse (OR 1.862) and three domains of quality of life such as physical health, psychological health and social relationship (OR 0.942, 95% CI 0.908-0.978; OR 0.955, 95% CI 0.916-0.995; OR 0.933, 95% CI 0.894-0.973 respectively).</div>\r\n<div>\r\n	Logistic regression analysis of the deemed significant variables however, did not show any significant association. This could be due to small sample size.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Even though there were no significant association based on binary logistic regression, there is no doubt that many confounders were operating which were masking associations. The prevalence of sexual dysfunction among married gynaecological cancer patients was 65%. This result was consistent with other studies done worldwide which stated the range from 40 to 100% (Anderson 1990; Casey 1996; Taylor 2000). Consistent with a study by Schover et al. (1998), the most common sexual problem for gynaecological cancer patients was sexual desire dysfunction. Gynaecological cancer patients are often able to reach sexual orgasm. However, it may be delayed due to cancer treatment, depression or anxiety (Schover 1997). Sexual orgasm dysfunction had the least prevalence, sharing the same prevalence as sexual pain disorder. According to studies (Schover 1997; Ganz et al. 1998; Broeckel et al. 2002), many sexual problems, even under favorable circumstances, may not resolve within the first two years of disease-free survival or may remain constant or even continue to increase. This could possibly explain that married cancer patients in this study with absence of sexual intercourse for more than one month accounted for more than half (53%) and there was association between this and sexual dysfunction.</div>\r\n<div>\r\n	The present study indicated that those with low education level experienced more sexual dysfunction. This might be due to the lack of understanding of their sexual difficulties and resources in getting treatment that could allay their sexual problems.</div>\r\n<div>\r\n	In the present study, patients with shorter cancer duration had higher prevalence of sexual dysfunction. Initial phase of diagnosis can be more distressing to some patients because they may experience direct or indirect sexual side effects of treatment. They are worried that sexual intercourse will further injure their diseased sexual organ or they may have cancer recurrence as long as the survival period is not over. The experience of the pain makes them handicap while performing sexual acts and cause emotional and spiritual disturbances which result in sexual dysfunctions.</div>\r\n<div>\r\n	Ongoing chemotherapy was associated with sexual dysfunction. This could be due to the direct and indirect side effects of the chemotherapy that cause the sexual dysfunction (Amsterdem &amp; Krychman 2008; Schover 1997; Anderson 1999; Auchincloss et al. 1998).</div>\r\n<div>\r\n	In the present study, there was no association between sexual dysfunction and major depressive disorder. This could be explained that when life threatening cancer became the major concern for the cancer patients, they were less disturbed by the sexual dysfunction. They might be so depressed that sexual problem was not as problematic as the depression itself. The distress due to sexual problem might become concern only after survival of cancer or stability of health was restored or guaranteed. This could be after 3 to 5 years of its diagnosis. Other possible explanation was that the cancer patients in their reproductive age in this study were minority (18%) where sexual intercourse might not serve the important purpose of reproduction. It was also possible that patients who had sexual problem might attribute it to their menopausal syndrome which they had to experience one day, they might not be overly concerned about its effect on sexual problems. Another possibility was that these women might also view sexual problem as a commonly shared problem among gynaecological cancer patients and thus became less distressed.</div>\r\n<div>\r\n	Cancer patients who had sexual dysfunction obtained lower scores in the physical health, psychological health and social relationship domains of quality of life. Sexual dysfunction and poor quality of life are bothersome to many cancer patients. This may interfere with a return to normal post-treatment life. Assessment, referral, intervention, and follow-up are mandatory for improving sexual dysfunction to maximize quality of life (Ganz et al. 1998).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Sexual dysfunction is highly prevalent in gynaecological cancer patients who were married and various risk factors were also involved.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1202, 1194, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	World Health Organization (2012) reported that at least one in every ten adults die everyday due to smoking induced diseases. This is equivalent to seven people dying every minute, which translates to about 4 million mortalities, worldwide. Globally, approximately 80% and 40% of lung cancers and chronic respiratory disease, respectively, are caused by smoking. To add, around 2.5 million deaths worldwide are related to harmful use of alcohol. Of these total deaths, over 300,000 happen in youngsters, accounting for 9% of all types of mortality in that age cohort (World Health Organization 2011a). Moreover, World Health Organization (2011a) reported that about 22% of cardiovascular diseases and 20% of cancers in the world are caused by alcohol.</div>\r\n<div>\r\n	In today&rsquo;s fast paced and busy lifestyle, smoking and alcohol consumption have become a serious public health concern in Malaysia. Each year, about 20% of total mortalities in Malaysia are associated with smoking (Lim et al. 2009; Tan et al. 2009b). This translates to approximately 10,000 deaths in the nation. In 2006, about one fourth of hospitalisations in Malaysia were associated with smoking related diseases (Lim et al. 2009). With regard to economic burden, Malaysian government spends more than RM 3 billion yearly on treating smoking induced diseases, such as, lung cancer, cardiovascular diseases and chronic respiratory diseases (Tan et al. 2009b).</div>\r\n<div>\r\n	In terms of alcohol, Malaysia was among the top rankings in alcohol consumption in the world in 2011, which had high per capita alcohol consumption (World Health Organization 2011b). Each year, Malaysian adults spend a total of USD 500 million on alcohol (World Health Organization 2011b). Besides, alcohol is also a leading factor causing poverty in Malaysia, as a rural worker may spend all his monthly individual income (approximately USD 80) just on alcohol (Tan et al. 2009a). Worse still, the Road Safety Council of Malaysia reports that at least three out of every ten road accidents throughout the country are related to alcohol consumption (World Health Organization 2004; Tan et al. 2009a).</div>\r\n<div>\r\n	There is a growing literature investigating the socio-demographic determinants of smoking and alcohol drinking in western and well-developed countries (Zhao &amp; Harris 2004; Manrique &amp; Jensen 2004; Aristei &amp; Pieroni 2008; Bilgic et al. 2010; Lin 2010; Yuan &amp; Yen 2012; Redonnet et al. 2012). The studies have consistently found that age, income, gender, marital status and education are significantly correlated to the likelihood of smoking and alcohol consumption. It is, however, that the literature is nearly silent on whether there are socio-demographic differences in smoking and alcohol drinking in Malaysia, where chronic diseases are prevalent.</div>\r\n<div>\r\n	Although Tan et al. (2009a) (2009b) have investigated the socio-demographic determinants of smoking and alcohol consumption in Malaysia, the studies appears to have several limitations. First, analysis of the studies is done by using household expenditure data, which only possesses information about household head&rsquo;s socio-demographic profile and the aggregate amount of tobacco and alcohol products purchased by the household. In fact, use of individual health survey data may seem more suitable than household data because it provides more detailed information about individuals&rsquo; smoking and drinking behaviours. Second, the studies do not take account of hereditary factor for analysis. Since, presence of hereditary illnesses may affect individuals&rsquo; health awareness, studies that neglect such factor may seem incomprehensive.</div>\r\n<div>\r\n	The objective of the present study was to fill this research void by undertaking an in-depth investigation on the socio-demographic determinants of smoking and alcohol drinking among Malaysian adults. Two main research questions were addressed&ndash;(1) what socio-demographic factors have a significant effect on individuals&rsquo; decision to smoke and drink alcohol? and (2) what policy can be proposed based on the findings of the present study? Overall, the present study creates two substantial contributions to the existing literature. First, the focus of the present study is on a developing country, Malaysia, where only few studies exist. Second, in addition to socio-demographic variables, the present study includes a hereditary variable (i.e. history of serious family illnesses) with the aim of examining its impact on smoking and alcohol drinking.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	DATA</div>\r\n<div>\r\n	Penang is a state in Malaysia located on the north-west coast of Peninsular Malaysia. Among all the states in Malaysia, the prevalence of alcohol consumption in Penang is the highest, placing it the fourth highest alcohol consumption state in Malaysia in 2011 (Institute for Public Health 2011). Besides, Penang is also regarded as one of the most developed states in Malaysia (The Star 2012), where people often live a hectic and unhealthy life. Given these reasons, it seems worthwhile to base the present study on Penang.</div>\r\n<div>\r\n	A cross-sectional primary survey data of Penang was used in the present study. Owing to time, budget and geographical constraints, the data was collected based on a non-probabilistic convenient sampling approach. Nonetheless, efforts were made to stratify the sample in proportion to the ethnic structure of Penang population (41.6% Malays, 40.9% Chinese and 17.5% Indian/others) (SERI 2011). Several public settings in the urban areas of Penang Island, such as, universities, streets, shopping malls, offices and cafes, which comprised various ages, income, races and education levels of individuals were selected to carry out the survey. The survey period was from August to October 2010. The inclusion criteria were those aged 21 years old and above, and who resided in Penang for no less than 12 months. Ethical approval was obtained for this study.</div>\r\n<div>\r\n	Piloted bi-lingual (Bahasa Malaysia and English) questionnaires were distributed to the selected respondents for self-administration. Nevertheless, some brief explanations were provided by the interviewers upon giving out the questionnaires. During the survey, two main questions were addressed &ndash; (1) &ldquo;Did you smoke any cigarettes in the past 30 days?&rdquo; (2) &ldquo;Did you consume any alcohol in the past 30 days?&rdquo; Besides, the respondents&rsquo; socio-demographic and health profiles, such as, age, income, education, ethnicity, gender, marital status and history of serious family illnesses were also elicited. Based on 95% confidence level, the calculated target sample size was 400 respondents which represented 1,609,900 adults in Penang. The overall response rate of the survey was 99.50% (398 respondents).</div>\r\n<div>\r\n	DEPENDENT VARIABLES</div>\r\n<div>\r\n	The present study used &lsquo;smoking&rsquo; and &lsquo;alcohol consumption&rsquo; as dependent variables. Following the guideline of Institute for Public Health (2008), smoking referred to the respondents who smoked in the past 30 days preceding the survey, while alcohol consumption referred to the respondents who consumed alcohol in the past 30 days preceding the survey. The dependent variables were thus included in the current models as two categorical variables. For smoking variable, a value of 1 was coded if the respondents smoked and 0 otherwise, whereas, for alcohol consumption variable, a value of 1 was coded if the respondents consumed alcohol and 0 otherwise.</div>\r\n<div>\r\n	INDEPENDENT VARIABLES</div>\r\n<div>\r\n	Owing to the paucity of in-depth empirical studies examining the socio-demographic determinants of smoking and alcohol consumption in Malaysia, the independent variables of the present study were selected based on previous studies that were conducted elsewhere (Zhao &amp; Harris 2004; Manrique &amp; Jensen 2004; Aristei &amp; Pieroni 2008; Bilgic et al. 2010; Lin 2010; Yuan &amp; Yen 2012; Redonnet et al. 2012). In particular, the independent variables of the present study were: (i) age; (ii) gender; (iii) ethnicity; (iv) marital status; (v)income; (vi) education; and (vii) history of serious family illnesses.</div>\r\n<div>\r\n	Age was included as a continuous variable to allow for a linear relationship. Ethnicity was divided into three categories: Malay, Chinese and Indian/others. Marital status was grouped into two groups: married and unmarried (i.e. single/ divorc&eacute;/ widow). Following Cheah (2011), the respondents&rsquo; monthly individual income was segmented into four categories: low (RM999), lower-middle (RM1,000 &ndash; RM2,999), upper-middle (RM3,000 &ndash; RM5,999), and high (RM6,000). One USD is approximately 3.2 RM. Education was categorised into three categories: primary, secondary and tertiary. History of serious family illnesses referred to the respondents whose parents were diagnosed with chronic diseases, such as, cancers, hypertension, hypercholesterolemia, diabetes, heart disease and stroke.</div>\r\n<div>\r\n	STATISTICAL ANALYSIS</div>\r\n<div>\r\n	The present study applied logistic regression models to examine the factors affecting the odds of smoking and alcohol consumption among the respondents. Both Likelihood Ratio (LR) and Pearson &chi;2 tests were conducted to assess the goodness-of-fit of the regression models. In addition, correlation coefficients between income and education variables were also calculated in order to detect the potential multicollinearity problem. The level of significance of all the tests was based on p-value of less than 0.05 (two-tailed). A total of 398 respondents were used for analysis. The statistical analysis was performed using Stata statistical software (Stata Corp 2005).</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	CHARACTERISTICS OF THE SURVEY RESPONDENTS</div>\r\n<div>\r\n	Of the total respondents, 14.57% were smokers and 31.91% were alcohol drinkers. The average age of the respondents was approximately 37 years. Although, more than half (55.78%) of the entire sample were females, the majority of smokers (91.38%) and alcohol drinkers (62.99%) were males. The ethnic breakdown consisted of 37.94% Malays, 40.95% Chinese and 21.10% Indian/others. This ethnic distribution closely mirrors the structure of Penang population, which comprises 41.6% Malays, 40.9% Chinese and 17.5% Indian/others (SERI 2011) (Table 1).</div>\r\n<div>\r\n	The distribution of married (49.75%) and unmarried respondents (50.25%) was almost equal. The majority of the respondents were in the lower-middle income group (44.97%), followed by those in the low (32.16%), upper-middle (18.59%) and high income groups (4.27%). Also, the majority of smokers (46.55%) and alcohol drinkers (40.16%) were in the lower-middle income group. A large proportion of the respondents had tertiary education (64.82%), followed by those with secondary (30.40%) and primary education (4.77%). The majority of smokers had secondary education (50.02%), whilst a large percentage of alcohol drinkers had tertiary education (77.17%). About half of the respondents in the entire sample had history of serious family illnesses (50.50%).</div>\r\n<div>\r\n	LOGISTIC REGRESSION ANALYSIS OF SMOKING AND ALCOHOL DRINKING</div>\r\n<div>\r\n	In terms of goodness-of-fit, the LR&chi;2 for both smoking and alcohol drinking models had a p-value of less than 0.05. Hence, the null hypothesis can be rejected, and this concludes that both of the models were very well fitted. In addition, the Pearson &chi;2 for smoking and alcohol drinking models had a p-value of more than 0.05. Thus, the null hypothesis was unable to be rejected, further indicating that both of the models fitted the data well (Table 2 and 3). The correlation coefficients between income and education variables were less than 0.8, thus suggesting that there was no serious multicollinearity problem in the models (Studenmund 2006) (Table 4).</div>\r\n<div>\r\n	The results illustrate that the males had higher odds of smoking (OR: 26.678) and drinking alcohol (OR: 5.051) than the females. On one hand, Chinese (OR: 0.177) and Indians/others (OR: 0.331) were found to have a lower likelihood of smoking than Malays. On the other hand, Chinese (OR: 37.796) and Indians/others (OR: 10.863) had a higher likelihood of consuming alcohol relative to Malays. The present study found that married individuals had lower odds of drinking alcohol than unmarried individuals (OR: 0.380). In terms of education, individuals with tertiary education are found to have a lower probability of smoking than their counterparts with only secondary education (OR: 0.258).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The present study found that gender, ethnicity and education can significantly affect smoking. In particular, males, Malays and individuals with secondary education had a higher likelihood of smoking compared to others. In terms of alcohol, gender, ethnicity and marital status were found to have a significant influence on the likelihood of alcohol drinking. Specifically, males, Chinese, Indians, individuals from other ethnic backgrounds and unmarried individuals were more likely to consume alcohol than others.</div>\r\n<div>\r\n	The findings of the present study showed that gender plays a significant role in affecting smoking as males have a higher likelihood of smoking than females, which lends support to those of Bilgic et al. (2010) based on the Turkish Household Expenditure Survey and Lin (2010) using a nationwide panel data of Taiwan. Also, the present study found significant gender differences in alcohol consumption as males were more likely to consume alcohol than females, which is consistent with the findings of previous studies (Manrique &amp; Jensen 2004; Yuan &amp; Yen 2012; Redonnet et al. 2012). The differences in risk and social preferences across gender may be a plausible explanation for these outcomes (Croson &amp; Gneezy 2009). In actual fact, females are more risk averse and socially sensitive than males. Since smoking and alcohol consumption are risky health behaviours, women tend to avoid them. Furthermore, smoking and alcohol consumption by females are less acceptable to the society than males (Cheah 2012). Thus, females who are sensitive to social cues, are less likely to smoke and drink alcohol than males. In light of these phenomena, public health authorities are suggested to pay special attention to reducing smoking and alcohol drinking among males. For example, public health authorities can use nationwide health awareness campaigns to advertise the information about the harmful effects of smoking and alcohol drinking on men&rsquo;s health, such as, erectile dysfunction and low sperm count.</div>\r\n<div>\r\n	The present study found a significant impact of ethnicity on smoking and alcohol drinking, which supports the findings of previous studies conducted in Malaysia (Tan et al. 2009a; Tan et al. 2009b; Tan 2012; Cheah &amp; Naidu 2012). Comparing among the ethnic groups, Malays were found to have the highest likelihood of smoking, whereas Chinese have the highest propensity to consume alcohol. This is likely attributable to the cultural and socioeconomic differences across ethnic groups. For instance, Malays (Muslims) in Malaysia are strictly prohibited from consuming alcohol because of their Islamic religious background, whereas Chinese often incorporate alcohol in their traditional festivals and celebrations. The main implication of this finding is that public health authorities should focus primarily on Malay and Chinese ethnic groups. As a measure towards reducing smoking and alcohol drinking among Malays and Chinese, respectively, a successful policy should use spokespersons from Malay and Chinese ethnic groups, as well as language-based mass media, such as, radio, newspaper and magazine to highlight the adverse effects of smoking and alcohol consumption on health.</div>\r\n<div>\r\n	In line with the findings of Zhao and Harris (2004), married individuals are less likely to consume alcohol than unmarried individuals. The study drew on the Australian National Drug Strategy Household Surveys to explore the factors associated with household purchase decisions of several types of drugs, such as, marijuana, alcohol and tobacco, and found married individuals to be 1.1% less likely to consume alcohol than single individuals. A plausible factor that can explain such finding is that married individuals have more family commitments than their unmarried counterparts, and also often carry more responsibilities to look after their family (Cheah 2013). Hence, they are generally more aware of their health and the risks of unhealthy lifestyle than unmarried individuals. Besides, married individuals also tend to play a good healthy role model as they do not want their children to engage in unhealthy behaviours.</div>\r\n<div>\r\n	Education is found to be significantly correlated to smoking. This finding is consistent with those of Yen (2005), Aristei &amp; Pieroni (2008), Bilgic et al. (2010) and Lin (2010) that higher educated individuals are less likely to smoke than lower educated individuals. Perhaps, this is because education can improve health by enhancing individuals&rsquo; health knowledge (Grossman 1972; Kenkel 1991). Well-educated individuals tend to be aware of the harmful effects of smoking on health, thus, they are likely to avoid smoking. Another plausible rationale is that a high level of education is associated with a low rate of time preference (van der Pol 2011). Individuals&rsquo; with a lower rate of time preference tend to be more patient and future oriented than individuals&rsquo; with a higher rate of time preference, and consequently have a lower likelihood of indulging in unhealthy behaviours (van der Pol 2011). In terms of policy implication, government should urgently introduce a nationwide health education programme with a focus on the less-educated segment of the population. In particular, the programme should provide workshops and seminars to educate the public about the risks and disadvantages of smoking.</div>\r\n<div>\r\n	Given the budget, time and geographical constraints, the present study had two inherent limitations. First, the collected sample could not represent the Malaysian populations as a whole. Hence, the findings may not be able to be generalised to the entire country. Second, several important variables, such as, household size and employment status could not be included for analysis. A future study is, therefore, suggested to seek data that can overcome these limitations when examining the factors affecting smoking and alcohol consumption in Malaysia.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Drawing on a cross-sectional sample of adults in Penang (Malaysia), the present study has thrown new light on the factors associated with smoking and alcohol drinking. The present study has found socio-demographic factor to be able to influence individuals&rsquo; decision to smoke and drink alcohol. The findings of the present study appear to have an important implication for the Ministry of Health Malaysia in terms of formulating population-based intervention measures.</div>\r\n', 1),
(1205, 1197, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Cerebral palsy (CP) is a group of permanent non-progressive disorder of movement and posture causing limitation of activities. It is attributed to the non progressive disturbances of the fetal or infant developing brain. Motor disturbances occur in combination with disturbances of sensation, perception, cognition, communication and behavior. Epilepsy and secondary musculoskeletal problems may co-exist (Rosenbaum et al. 2007). Microcephaly, drooling and varying degree of mental retardation are other manifestations (National Institute of Neurological Disorders and Stroke 2013). Symptoms are usually detected in infancy when a child presents with delayed milestones of development.</div>\r\n<div>\r\n	The prevalence of CP differs from country to country ranging from 2.0 to 2.5 per 1000 live birth (Reddihough &amp; Collins 2003). Many causes have been implicated for CP such as congenital and environmental factors. These include malformation of the brain, intrauterine infections, low birth weight and birth asphyxia. Some studies have hypothesized genetic factors or a combination of genetic and environmental factors as the underlying cause for CP (Gibson et al. 2008; Djukic et al. 2009).</div>\r\n<div>\r\n	Familial CP or recurrent CP among siblings has been described in the literature (Rajab et al. 2006; Mukherjee et al. 1973). However recurrent CP among non-consanguineous siblings is rare. We describe an unusual case of two male siblings from a non-consanguineous marriage affected by familial CP. It is hoped that this case study increases the awareness on familial CP so that early detection, genetic counseling, appropriated health education, and care can be provided for the affected families.</div>\r\n<div>\r\n	Informed consent for the publication of patient&rsquo;s information and images used in this article was obtained from the parents.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	Two male siblings of Indian ethnicity, aged 22 (sibling A) and 20 years (sibling B) were identified to be affected by CP. Patient A, the eldest son was diagnosed to have CP at the age of three while his younger brother, sibling B, was diagnosed to have CP at the age of two years. The third and youngest male sibling, currently 11 years old is physically and mentally normal. All three children were delivered at term without any significant antenatal, intra-partum and post-partum events or complications. Maternal age at first, second and third delivery was 26, 28 and 37 years, respectively.</div>\r\n<div>\r\n	Sibling A is the eldest of the three. He was delivered at term through spontaneous vaginal delivery with a birth weight of 2.8kg. The antenatal and birth history was uneventful except for congenital talipes equinovarus (CTEV) which was diagnosed at birth. Parents noted a delay in milestones of developments compared to his peers, however they postponed professional advice until he was three years of age, hoping for spontaneous improvement. He could barely walk at the age of three years. He was diagnosed as CP based on global developmental delay of milestones and presence of spastic motor features. At the age of four years, he developed seizures and was started on anti-epileptics. He is currently 22 years of age, has dysmorphic features such as microcephaly, microngathia, drooling of saliva and dysarthria. He also has surgical scars over both ankle and calves from the corrective CTEV surgery. He can walk short distances with an ataxic gait, able to scribble and feed himself with minimal help. He can indicate his toilet needs however, requires assistance from his mother who is his main caregiver. His speech is slurred and limited to single words and incomprehensible sounds. Physically, his lower limbs appear smaller and underdeveloped compared to his upper limbs. Neurological examination of the lower limb shows increase tone with a power of 4/5 bilaterally. All reflexes are brisk. Babinski&rsquo;s sign is up-going bilaterally. Upper limbs demonstrate occasional clumsy and non purposive movements.</div>\r\n<div>\r\n	Sibling B is the second born in the family and he is two years younger than sibling A. He was diagnosed to have CP at the age of two years based on global developmental delay and seizures. Currently, he has features similar to his elder brother which include dysmorphism, microcephally, microngathia, drooling of saliva and dysarthria. However, his gait is more stable as he does not have CTEV. He is able to feed himself with minimal assistance. His speech is slurred and consists of incomprehensible sounds. Neurological examination of the lower limb demonstrates increase tone, normal power and brisk reflexes. His hands show sudden, non purposive movements but to a lesser extent compared to his elder brother. In summary, both affected siblings have microcephaly, spastic diplegia, dysarthria, seizure and mental retardation. Their social skills are equivalent to a three year old child. Figure 1 shows microcephaly and the dysmorphic features of these two boys.</div>\r\n<div>\r\n	Their father who is now 53 years old, works with the local municipal council. His mother, aged 48 years is a homemaker and cares for her two disabled children. Both parents are physically and mentally normal. Both have type II diabetes, diagnosed five years ago and maintain good glycaemic control with medications. There is a strong family history of epilepsy among the paternal cousins. Two male siblings are affected by epilepsy while another cousin is mentally challenged. However, it could not be confirmed if any of them had features CP. Figure 2 depicts their family genogram.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Although rare, familial CP has been reported as early as the 1960s among Jewish population where consanguineous marriages are a norm, suggesting a possible genetic inheritance (Adler 1961). Familial CP has also been reported in Asian regions where consanguineous marriages are a common practice. A case report from Oman documented 10 out of 44 members from a large family of consanguineous marriage to be affected by CP suggesting a genetic form of inheritance for CP (Rajab et al. 2006). The familial CP described by Rajab had common features such as spastic diplegia, microcephaly and mental retardation suggesting a possible recessive mode of genetic inheritance for CP.</div>\r\n<div>\r\n	Familial CP arising from a non-consanguineous union is rare. In Sweden where consanguinity is uncommon, familial CP accounts for about 1.6% of all cerebral palsy cases. A study by Hemminki et al. (2007) found that parents who had had one affected child with CP were at risk of recurrence. They identified a 4.8 fold risk of having a second child with CP when the first was affected and increases to 29 folds if the siblings were twins (Hemminki et al. 2007). CP was also found to occur more frequently in multiple pregnancies compared to singleton pregnancies (Pharoah 2005). In another observation, Richer et al. (2011) suggest that recurrent CP among siblings from non-consanguineous parents could be due to a complex combination of maternal and genetic factors (Richer et al. 2011).</div>\r\n<div>\r\n	Recent data from a large Norwegian cohort study also suggests the possibility of a strong genetic influence. The interaction between multiple genes and the environment is postulated as the underlying cause for CP (Toll&aring;nes et al. 2014). McHale et al. (2000) identified a possible genetic link among Asian siblings affected by a similar type of ataxic CP to the involvement of chromosome 9p12&ndash;q12 (McHale et al. 2000).</div>\r\n<div>\r\n	In summary, this case report depicts two male siblings who are products of a non-consanguineous marriage affected by CP. Both parents were healthy at time of conception and mother was well throughout both pregnancies. Intrapartum and postpartum events were unremarkable. These siblings share common features of CP such as ataxia, microcephaly, spastic diaplegia, epilepsy and mental retardation. Family history of mental retardation and epilepsy in the paternal lineage suggests that paternal genetic factors may have a contributory role in determining familial CP. Current evidence from the literature suggests a recessive or a more complex mode of inheritance for familial CP which may only be unraveled by further research in this area. The paucity of investigations such as early MRI of the brain to study the neuronal formation and genetic studies, at the time of diagnosis of CP are some setbacks of this case. Genetic studies were not performed due to the lack of facilities and high cost involved in performing such tests. The diagnosis of CP necessitates family counseling to discuss the risk of recurrences. Early rehabilitation should also be initiated as familial CP predicts poor prognosis and low rehabilitation success rates (Adler 1961).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The recurrence of CP among siblings from non-consanguineous parents and single births such as this case, illustrates a possible complex genetic mode of inheritance. Although this form of CP is uncommon, the consequence of having multiple siblings affected by CP would have a major impact to the family and community in general. Physicians should be aware of the familial type of CP and refer to pediatricians for genetic screening. Counseling should be provided to parents with one child affected by CP of the possible risk of recurrence. Consanguineous unions in general, should be discouraged to reduce the risk of recurrent CP and other autosomal recessive conditions. Since a definite genetic mode of inheritance is yet to be established for CP, it is a potential area for future research which may bridge the gaps in knowledge pertaining this complex condition.</div>\r\n', 1),
(1211, 1203, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	DM and HD have become an increasingly significant public health concern and the prevalence of which is increasing globally in both developed and developing countries like Asia, Africa and Europe (Abbott et al. 2005). Diabetes contributes to about more than 60% of the world&rsquo;s diabetic population and leprosy accounts to about 189,018 for the first quarter of 2013 in Asia and Africa (Ramachandran et al. 2012). Complications arises from lower limb in persons with DM and HD ranges from peripheral vasoconstriction, ischemia, neuropathy which subsequently leads to foot ulcers and infections (Wu et al. 2007). These ulcers continue to cause great morbidity in these special populations. Hence, the care for the ulcers of the plantar aspects requires multi disciplinary approach which will improve subject&rsquo;s functional capacity and quality of life (Frykberg 2002). Recommended management of these ulcers includes foot assessment, intensive education, therapeutic shoes, padded stockings, infection control, debridement, pressure mitigation, total contact cast, Ultrasound therapy, Laser, cryotherapy and exercises (Potturi &amp; Reddy 2014).</div>\r\n<div>\r\n	Although many treatment options were put forth for treating the ulcers, total contact casting for promoting healing proved to be an effective method. Nevertheless, the techniques of application and the materials vary in each and every study. Apart from that, limited experience in plaster boot cast application among the region has suggested that total contact plaster boot may resolve ulcers. Hence, keeping in view of difference in techniques and cost effectiveness, we have designed a modified methodological approach of applying this boot over a series of patients who are indulged with ulcers via off loading method using soft padding. This case series, report the results obtained in a group of subjects with foot ulcers treated with total contact plaster boot in a rural based institution.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Subjects who are diagnosed with non-healed plantar ulcers with age range between 40 to 60 years old were recruited to participate in this study. Those, who are diagnosed to have HD and DM with non-healed plantar ulcers, were recruited for the study and those who had infected ulcers, visual problems that could affect balance, an ulcer on the contra lateral foot were excluded from the study (Faglia et al. 2010). The study protocol was approved by institutional ethics committee. A total of 13 potential subjects were screened. Three subjects were excluded as they didn&rsquo;t meet the criteria set for the study. Thus, 10 subjects were included in the study. Following screening for infective pathology, subjects consentwas obtained and they were admitted to Division of Physical Medicine and Rehabilitation (DPMR), Rajah Muthiah Medical College &amp; Hospital (RMMC &amp; H), Annamalai Nagar, India.</div>\r\n<div>\r\n	PROCEDURES: MEASUREMENT OF WOUND</div>\r\n<div>\r\n	Initially, demographic details such as age, sex were recorded in the data recording sheet which was prepared for the present study. Then details on duration of diabetes, ulcer characteristics were recorded as shown in Table 1. The measurement of wound size was traced by placing a tracing paper over the wound and it was reinforced with another tracing paper. Subsequently, a pencil was used to trace the circumference of the wound. After marking, the tracing paper which was kept over the wound, the tracing paper was discarded to prevent infection. Then the reinforced tracing paper was kept for further accomplishment. The wound surface area was calculated by using (Area = length X breadth of the wound). Length was measured between 12˚ to 6˚ clockwise directions and breadth was measured between 3˚ to 9˚ clockwise directions which is the widest part perpendicular to the length (Srivastava &amp; Dugaprasad 2008).</div>\r\n<div>\r\n	After measurement of wound, the subjects were prepared for casting. Initially they were positioned in supine lying, the wound was cleaned properly and soft cotton padding was kept over the wound as a source of off loading, then the plaster boot was applied from head of metatarsal to ankle joint with the help of plaster of Paris. Appropriate care was taken such that the plaster did not disturb the ankle movements. After completing the casting procedure, the subjects were discharged and are advised to report 15 days following plaster cast application. Instruction and pamphlets related to the study were given to report immediately to the hospital when they encounter signs of fever, lymphadenopathy and foul smell from the wound. Subjects were advised to cover the plaster boot with the help of plastic bag when necessary to prevent wetting.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Totally ten subjects were enrolled and both genders contributed equally to the study with a mean age of 48.4 years old. Almost 40 percent of the subjects had ulcers in 1st metatarsal region and 30 percent had ulcers in the region of heal. Comparison of pre and post test values of wound size area was computed using paired&lsquo;t&rsquo; test and the statistical significance was kept at the level of 0.5. The comparisons of pre and post test measures of wound size area are shown in Table 2. Results showed that there was significant difference between pre and post test values of wound size area (p&lt;0.05), which shows that total contact cast boot promoted wound healing among these populations.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The present study found out that total contact plaster boot applied to plantar ulcers of the foot via off loading methods promotes wound healing. The results of this study will now be compared to the findings of previous work. The present study are consistent with the findings of an earlier study which was carried out on off loading methodology on diabetic foot comparing total contact casting (TCC), removable cast walkers (RCW) and half-shoes in which they have concluded that TCC appears to heal a higher proportion of wounds in a lesser duration of time than two other widely used off-loading modalities, the RCW and the half-shoe (Armstrong et al. 2001).</div>\r\n<div>\r\n	Similarly, another randomized controlled trail which was carried out in the year 2010, on forty five patients on removable and non removable cast concluded that removable cast walker was equivalent in efficacy to the TCC in terms of ulcer reduction. This study has similar results to our study in which TCC was used in two groups of populations for reducing the ulcer (Faglia et al. 2010).</div>\r\n<div>\r\n	The present study utilized plaster of Paris materials for casting. However, the materials and the techniques followed in our present study are different from the studies in which fibreglass bandages and other sort of materials were used to cast the feet. A systematic review published in the year 2013 concluded that non-removable off-loading device promotes better healing than removable off-loading devices (Morona et al. 2013).</div>\r\n<div>\r\n	These findings further support the idea of application of TCC for DM and HD foot ulcer using plaster of Paris which is cheaper and cost effective when compared to other materials utilized in other studies. The results of our study indicate that TCC applied through an easily available material like plaster of Paris in a rural set up where the series of cases were treated, proved to be an effective option in managing a complication like trophic ulcers of the feet for both DM and HD.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The fact designates that the application of total contact plaster boot is valuable in the administration of both Diabetic and Hansen&rsquo;s disease trophic ulcers of the feet.</div>\r\n<div>\r\n	ACKNOWLEDGMENT</div>\r\n<div>\r\n	The authors are grateful to the subjects who contributed for the study.</div>\r\n', 1),
(1215, 1207, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	In general, cosmetic breast augmentation with silicone implants is a safe procedure. It is associated with low surgical complications such as infection,&nbsp;abscesshaematoma and wound dehiscence. Late infection of breast implant is rare, but if it does occur can cause a lot of issues especially to the surgeon in term of establishing the diagnosis, treatment options ranging from conservative treatment with antibiotic alone to surgical intervention. We present a case of peculiar late breast implant infection which involved large area of anterior and lateral abdominal wall area that eventually underwent massive wound debridement with good outcome.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 33-year-old lady with a history of bilateral breast augmentation using silicone implants 15 years earlier, presented to the emergency department with complaint of left flank swelling, pain and fever for four days duration. The swelling was fluctuant, tender and erythematous with a small necrotic patch. It extended from the lower chest wall on the left to the lumbar region on the same side. She remained hemodynamically stable but her infective parameters such as leucocyte count and c- reactive protein were markedly elevated. CT scan of the thorax and abdomen showed significant tissue streakiness in the left breast suggesting ruptured left breast implant with subcutaneous collection extending from the left chest wall to the anterior superior iliac spine and para-spinous region posteriorly. Emergency incision drainage and removal of the breast implant was undertaken with 1.9L of purulent material drained. Two separate incisions were made, one at the inframammary fold and a separate incision at the most dependent area of the swelling over the left flank region. Corrugated drains were placed in the abscess cavities after thorough debridement and irrigation. Histopathological examination of the silicone implant revealed foreign body granuloma, while microbiology culture of the purulent material was positive for Staphylococcus Aureus. Post operatively Cloxacillin was administered according to culture sensitivity report and subjected to daily dressing with saline. Five days later, the corrugated drains were removed and the patient was discharged home. She has since been followed-up in the surgical outpatient clinic and has shown full recovery. She is keen for insertion of a new implant on the same side, and she is presently awaiting her operation date.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Breast augmentation has become widely popular with increasing emphasis on achieving a more sexually appeasing figure. In 2010, breast augmentation was the most opted for cosmetic surgery in America with almost 320,000 women reported to have undergone surgical enhancement (American Society of Plastic Surgeons 2012).</div>\r\n<div>\r\n	Similarly in Asia, there is also a rise in trend of breast augmentation especially among young women below the age of 19. This global hunger for fuller breasts has prompted the need to question and establish the bio-durability of silicone implants. Although the exact bio-durability of silicone implants remains largely a mystery, previous studies have reported 10-year rupture rates to vary between 10-50% (Holmich et al. 2003; Heden et al. 2006; Thorton et al. 1988). This would mean that women are likely to require implant removal or change of implant at least once in their lifetime, if not more.</div>\r\n<div>\r\n	Infection of silicon breast implants is a rare yet the most dreadedcomplication with 1-2% of reported incidence. Skin commensals such as Staphylococcus Aureus, Lactobacillus, and Alpha-haemolytic Streptococcus have have been implicated as common causative organisms in the immediate post-operative setting (Brown et al. 2000; Thorton et al. 1988). Delayed presentation has been attributed to secondary bacterial infection from invasive procedures distant to the breast tissue or peri-prosthetic infection from infection of the breast tissue itself leading to strong opposition from surgeons against procedures such as nipple piercing in patients with breast implants in-situ. Prosthetic contracture or rupture of implant has also been associated with infection and is often seen after many years following surgery. Cause of implant rupture besides being largely idiopathic, has also been reported following breast compression during routine mammography, and trauma (Brown et al. 1997). Optimum placement of implant in relation to maximizing its bio-durability and minimizing morbidity is an ongoing debate. Although submuscular placement of silicone implant has been attributed to increased hematoma formation, there has not been any statistical significance reported between subglandular or submuscular placement in relation to infection or implant rupture (Hand et al. 2010). Women with silicone implant contracture are often asymptomatic and incidentally diagnosed during mammography. However, there has been an increase in evidence to link underlying subclinical prostethic infection with development of contracture as late as 9 years following initial surgery (Pajkos et al. 2003; Virden et al. 1992). There is currently no consensus on removal of contracted silicone implant in asymptomatic women, although surgery is the preferred mainstay of treatment for Baker&rsquo;s grade 3 and beyond.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The traditional approach to management of infected silicone breast implants has always been removal of the offending prosthesis, treatment of the underlying infection with appropriate antibiotics and placement of a new implant at a later date. The advents of stronger antibiotics in addition to more accurate microbiology culture and sensitivity reports have paved the way for an alternative method of treatment. With careful patient selection, mild prosthetic infection canbe treated non-surgically with antibiotic therapy alone for 10 to 14 days followed by clinical and radiological reassessment. Removal of implant is reserved for moderate to severe form of infection, and failure to respond to antibiotic therapy (Spear &amp; Seruya 2010). In our patient, the decision for debridement and removal of implant was made in view of severe ongoing infection involving a significant portion of chest wall and anterior abdominal wall. Subsequent decision to replace the removed silicone breast implant should depend on patient&rsquo;s preference, overall breast tissue condition and effectiveness of previous surgical and antimicrobial therapy (Pittet et al. 2005).</div>\r\n', 1),
(1221, 1213, NULL, NULL),
(1227, 1219, '<div>\r\n	MicroRNAs belong to one of the classes of non-coding RNAs. Non-coding RNAs are functional RNAs that do not translate into protein. They comprise: transfer RNA (tRNA); ribosomal RNA (rRNA); small nucleolar RNA (snoRNA); microRNA (miRNA); small interfering RNA (siRNAs); small nuclear RNA (snRNA); piwi-interacting RNA (piRNA), and long ncRNA. To date, there are approximately 1881 pre-miRNAs and 2588 mature human miRNAs have been identified (miRBase, June 2014). MicroRNAs are about 19-25 nucleotides in length and are now known to have important post-transcriptional roles in almost every cellular process in eukaryotes. These processes include the regulation of developmental timing and signalling pathways, apoptosis, metabolism, myogenesis and cardiogenesis, brain development, and human pathologies like viral diseases, genetic disorders and cancer (Shi et al. 2008).</div>\r\n<div>\r\n	THE DISCOVERY OF RNA INTERFERENCE</div>\r\n<div>\r\n	Fire and colleagues first reported that the injection of double stranded RNA caused the degradation of mRNA encoded by the gene unc-22 of the small nematode Caenorhabditis elegans. They termed this effect RNA interference (RNAi) and showed that it was sequence specific and effective at concentrations far lower than the target unc-22 mRNA, indicating the involvement of amplification effect in this process (Fire et al. 1998).</div>\r\n<div>\r\n	At the same time, David Baulcombe&rsquo;s group (Hamilton &amp; Baulcombe 1999) discovered posttranscriptional gene silencing (PTGS) in plants. Both viral infection and transgenic expression in plants can induce PTGS, which targets both cellular and viral mRNA. It was inferred that PTGS operates through the generation of small RNA molecules of 21 to 25 nucleotides, which are now known as small interfering RNAs (siRNAs). Consequently, work on both animals and plants together revealed a highly conserved mechanism of RNA interference that had evolved at least in part for combating viral infection.</div>\r\n<div>\r\n	Subsequently, Philip Sharp group was able to recapitulate RNA interference in cell free extracts from Drosophila cells, which led to the establishment of three phases of the RNA interference reaction: cleavage of a long dsRNA into shorter dsRNA segments by Dicer; the loading of single stranded RNA into the RISC (RNA-induced silencing complex) and the targeting and degradation of mRNA by this complex (Tuschl et al. 1999). Subsequently mutations of C.elegans that confirm resistance to RNA interference were found to disrupt genes that encoded components of RISC (Fire 2007). Eventually, Andrew Z. Fire and Craig C. Mello became the recipients of Nobel Prize in Physiology and Medicine in 2006 (www.nobelprize.org). While David C. Baulcombe, together with Victor R. Ambros and Gary B. Ruvkun received the Albert Lasker Award for Basic Medical Research (www.laskerfoundation.org).</div>\r\n<div>\r\n	THE DISCOVERY OF MICRORNAS</div>\r\n<div>\r\n	Lee et al. (1993) identified two overlapping transcripts of the lin-4 gene of C. elegans, of approximately 22 and 61 nts that inhibited the expression of lin-14 through complementarity to the 3&rsquo; untranslated region (UTR) of lin-14 mRNA. The 61 nucleotides molecule can also fold into a double-stranded &ldquo;hairpin&rdquo;. They suggested that lin-4 inhibits translation of lin-14 through an antisense RNA-RNA interaction.</div>\r\n<div>\r\n	Subsequently, it was shown that lin-4 and a second gene let-7 acted in a sequential stage specific expression pattern that regulates the timing of C.elegans development. The let-7 gene encodes a 21-nucleotide RNA that is complementary to the 3&rsquo; UTR of genes lin-14, lin-28, lin-41, lin-42 and daf-12. Let-7 is expressed at the adult but not embryonic stage. Let-7 was also identified in humans, fruit flies, chickens, frogs, zebrafish, molluscs and sea urchins and the binding site in its target was conserved in some of these organisms (Pasquinelli et al. 2000). In 2001, there were discovery of large number of similar small RNA molecules, referred to as microRNAs, in C.elegans and subsequently mouse (Lee &amp; Ambros 2001).</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	I would like to express my sincere thanks to Nicholas Dibb, Imperial College London for his advise and suggestions.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1228, 1220, '<div>\r\n	PHARMACOVIGILANCE AND REPORTING ADVERSE DRUG REACTIONS</div>\r\n<div>\r\n	Drug therapy is an essential part of health care and has become a universally accepted form of medical treatment, both by health professionals and society at large. It is not an exaggeration to say that almost every human being has had an experience with drug therapy, even newborn infants are now exposed to immunisations on the day they are born. What does society want from drug therapy? Walley (1995) summarised the needs of the public as the &lsquo;3 E&rsquo;, efficacy, effectiveness and efficiency.</div>\r\n<div>\r\n	&bull; Efficacy: the drug works (fulfils its intended purpose) under optimal conditions, and is safe and tolerable</div>\r\n<div>\r\n	&bull; Effectiveness: the drug works under the conditions of everyday use</div>\r\n<div>\r\n	&bull; Efficiency: be defined as for every dollar spent, the greatest possible benefit is gained.</div>\r\n<div>\r\n	Pharmacovigilance deals with the first 2 Es of drug therapy; efficacy and effectiveness. Any substance capable of producing therapeutic effect also produces unwanted or adverse effects. It is therefore important to identify any adverse drug effect to eliminate or minimise morbidity and mortality due to drug use. ADRs harmfully affect patient&rsquo;s quality of life, increase cost of patient care and cause patients to lose confidence in their doctors and their treatment.</div>\r\n<div>\r\n	The year 1961, after the thalidomide disaster, that pharmacovigilance really took a foothold with huge international effort to address the issue of drug safety. An Australian obstetrician, William McBride reported an increase in foetal malformations with unique characteristics, later known as phocomelia, in the babies of mothers prescribed the drug thalidomide (Routledge 1998). Dissemination of information was relatively slow and by the time thalidomide was withdrawn, thousands of babies were born with phocomelia and other disabling congenital malformations. There was a huge public outcry and governments PHARMACOVIGILANCE AND REPORTING ADVERSE DRUG REACTIONS</div>\r\n<div>\r\n	Drug therapy is an essential part of health care and has become a universally accepted form of medical treatment, both by health professionals and society at large. It is not an exaggeration to say that almost every human being has had an experience with drug therapy, even newborn infants are now exposed to immunisations on the day they are born. What does society want from drug therapy? Walley (1995) summarised the needs of the public as the &lsquo;3 E&rsquo;, efficacy, effectiveness and efficiency.</div>\r\n<div>\r\n	&bull; Efficacy: the drug works (fulfils its intended purpose) under optimal conditions, and is safe and tolerable</div>\r\n<div>\r\n	&bull; Effectiveness: the drug works under the conditions of everyday use</div>\r\n<div>\r\n	&bull; Efficiency: be defined as for every dollar spent, the greatest possible benefit is gained.</div>\r\n<div>\r\n	Pharmacovigilance deals with the first 2 Es of drug therapy; efficacy and effectiveness. Any substance capable of producing therapeutic effect also produces unwanted or adverse effects. It is therefore important to identify any adverse drug effect to eliminate or minimise morbidity and mortality due to drug use. ADRs harmfully affect patient&rsquo;s quality of life, increase cost of patient care and cause patients to lose confidence in their doctors and their treatment.</div>\r\n<div>\r\n	The year 1961, after the thalidomide disaster, that pharmacovigilance really took a foothold with huge international effort to address the issue of drug safety. An Australian obstetrician, William McBride reported an increase in foetal malformations with unique characteristics, later known as phocomelia, in the babies of mothers prescribed the drug thalidomide (Routledge 1998). Dissemination of information was relatively slow and by the time thalidomide was withdrawn, thousands of babies were born with phocomelia and other disabling congenital malformations. There was a huge public outcry and governments worldwide especially within Europe and the United States recognised there was a need to regulate and monitor the safety of drug use. This led to the Sixteenth World Health Assembly (1963) which adopted a resolution for the rapid dissemination of ADRs information, and prompted the creation of the WHO Pilot Research Project for International Drug Monitoring in 1968 (World Health Organization 2002a). This pilot research became the basis for pharmacovigilance systems worldwide and later resulted in a WHO consultation meeting in 1971 which advocated the establishment of national centres for drug monitoring and provided the first guidelines for national centres (World Health Organization 2002a).</div>\r\n<div>\r\n	According to the World Health Organisation (WHO) pharmacovigilance is define as the science and activities (observational or post-approval scientific data gathering activities) relating to the detection, assessment, understanding and prevention of adverse effects or any other possible drug-related problems with the goals of identifying and preventing these problems to the extent possible (World Health Organization 2002a). The most important task of pharmacovigilance is to identify &lsquo;signals&rsquo; of drug safety problems as early as possible. A signal is defined by the WHO as &ldquo;reported information on a possible causal relationship between an adverse event and a drug, the relationship being unknown or incompletely documented previously&rdquo; (World Health Organization 2002b). A &lsquo;safety signal&rsquo; refers to a concern about an excess of adverse events compared to what would be expected to be associated with a product&rsquo;s use (Food and Drug Administration 2005). According to earlier reports, the ultimate goal of pharmacovigilance is to foster the rational and safe use of medicines, the assessment and communication of the risks and benefits of drugs on the market and educating and informing of patients (Uppsala Monitoring Centre 2000).</div>\r\n<div>\r\n	A new medicine must pass three hurdles before its approval by the national drug regulatory authority. Sufficient evidence is required to show the new drug to be (World Health Organization 2004):</div>\r\n<div>\r\n	&bull; Of good quality</div>\r\n<div>\r\n	&bull; Effective</div>\r\n<div>\r\n	&bull; Safe for the purpose or purposes for which it is proposed</div>\r\n<div>\r\n	Whereas the first two criteria must be met before approval, the issue of safety is less certain. Safety is not absolute and can only be judged in relation to efficacy and limits of acceptable safety. Frequently when a new drug is marketed, many of its known adverse effects including those describe within the summary of product characteristics (SPC) and in textbooks are adverse effects establish during the third phase of drug trial (out of 4). This knowledge is often incomplete especially in regards to frequency, mechanism and risk factors. Tests in animals are insufficient to predict human safety. There is also a possibility that rare yet serious adverse events (such as those occurring with a frequency of one in two thousands) will not be detected in the pre-registration development of the drug. Prior to drug approval for market, it will have to be exposed to a number of patients to detect its efficacy and toxicity. However, due to the limitation of number of patients exposed (ranging from as few as 500 to 5000), the condition of use differ from &lsquo;real world&rsquo; clinical practice, the limited duration of clinical trials and increasing pressure on drug regulators from the pharmaceutical industry to shorten the review time for new medicines, detection of these less frequent adverse events is almost nil (Table 1) (Strom 2006). A fatal ADR occurring in 1 in 2000 patients treated with a new drug will only be recognized after 6000 patients have been treated and observed, and this is only likely provided that the background incidence of such a reaction is zero or a causal association with the drug is clear (Strom 2006). Even if detected, the event will be incompletely described and understood because of too few events. In addition, it may also be difficult to use clinical trials to ascertain the safety profile associated with chronic exposure to any product, exposure in populations with co-morbid conditions or taking multiple medications. More information is also frequently needed for drug use in specific population groups such as children, pregnant women and the elderly due to homogenous sample populations in clinical trials. Drug-drug interactions are also frequently not assess with much detail during the drug development process.</div>\r\n<div>\r\n	It was the thalidomide disaster (a teratogen) which started the era of pharmacovigilance; with much irony however pharmacovigilance involving pregnant women has not developed significantly since the disaster occurred. Drug trials and research involving pregnant women have been almost non-existent resulting in a gap in knowledge regarding which drugs are suitable for use during pregnancy. ADRs affect everyone and children are not spared. It is reported that 9% of children experience ADRs while in hospital and that approximately 0.8% of children suffers a fatal outcome with ADRs. Both figures are likely to be an underestimate, as it is well recognised that ADRs are significantly under reported (Clarkson &amp; Choonara 2002) Over 50% of medicines used in children may not have been studied in this age group (MHRA 2013)</div>\r\n<div>\r\n	DEFINITIONS AND CLASSIFICATION OF ADRS</div>\r\n<div>\r\n	There are various definitions for ADRs and ADEs, some of which are widely used while others have been proposed. The conventional and widely accepted definitions are proposed by the Uppsala monitoring centre (UMC) and the world health organisation (WHO) (Uppsala Monitoring Centre 2000; Uppsala Monitoring Centre). In defining ADR/E, various other related terms are defined as follows:</div>\r\n<div>\r\n	&bull; A drug or medicine is a pharmaceutical product, used in or on the human body for the prevention diagnosis or treatment of disease, or for the modification of physiological function.</div>\r\n<div>\r\n	&bull; An adverse (drug) reaction is a response to a medicine which is noxious and unintended, and which occurs at doses normally used in humans for the prophylaxis, diagnosis or therapy of disease, or for the modification of physiological function. (Normal dose clause distinguishes adverse reactions from poisoning and this clause was later refined by Meyboom et al. (2000), to caution on patients experiencing an adverse reaction at normal dose but may indeed be a case of high/toxic dose because of impaired renal/hepatic excretion or other reasons. It is common for the term adverse effect to be used as synonyms for adverse reaction. Adverse effect is seen from the point of view of the drug whereas an adverse reaction from the point of view of the patient (Edwards &amp; Aronson 2000). Another commonly used definition for an ADR was put forward by Edwards and Aronson (2000), who define an ADR as &ldquo;an appreciably harmful or unpleasant reaction, resulting from an intervention related to the use of a medicinal product, which predicts hazard from future administration and warrants prevention or specific treatment, or alteration of the dosage regimen or withdrawal of the product&rdquo;. The Medicines and Healthcare products Regulatory Agency (MHRA) in the United Kingdom has a broader definition of an ADR &ldquo;as an unwanted or harmful reaction experienced following the administration of a drug or combination of drugs, which is suspected to be related to the drug&rdquo; (British Medical Association 2006). Unlike the WHO definition, the MHRA definition does not exclude overdose or drug misuse.</div>\r\n<div>\r\n	&bull; An unexpected adverse reaction is an adverse reaction, the nature or severity of which is not consistent with domestic labelling or market authorisation, or expected from characteristics of the drugs.</div>\r\n<div>\r\n	&bull; A side effect is any unintended effect of a pharmaceutical product occurring at doses normally used in man, which is related to the pharmacological properties of the drug.</div>\r\n<div>\r\n	&bull; A toxic effect is an adverse effect of a drug which occurs as an exaggeration of the desired therapeutic effect and which is not common at normal doses (Edwards &amp; Aronson 2000). It is always dose-related.</div>\r\n<div>\r\n	&bull; An adverse event or experience is defined as any untoward medical occurrence that may present during treatment with a medicine but which does not necessarily have a causal relationship with the treatment.</div>\r\n<div>\r\n	&bull; Serious (not synonymous with &lsquo;severe&rsquo; which is used to describe the intensity of a specific outcome) adverse events/reactions can be defined as those that:</div>\r\n<div>\r\n	&bull; are life threatening or fatal</div>\r\n<div>\r\n	&bull; cause or prolong hospital admission</div>\r\n<div>\r\n	&bull; cause persistent incapacity or disability</div>\r\n<div>\r\n	&bull; concern misuse or dependence</div>\r\n<div>\r\n	&bull; Efficacy is the ability of a drug to produce the intended effect as determined by scientific methods, for example in pre-clinical research conditions.</div>\r\n<div>\r\n	RECOGNISING ADRS</div>\r\n<div>\r\n	Without the ability to recognise ADRs, it becomes meaningless to define ADRs. Recognising ADRs is not clear-cut as ADRs may act through the same physiological and pathological pathways as the disease being treated. A patient&rsquo;s drug history should include details regarding illicit drugs, herbal and homeopathic medicines, detailing any suspected drug-drug interaction and cross-reactivity. Concurrent viral infections may also increase the risk of ADRs; infectious mononucleosis increases the risk of rash in patients given amoxicillin by a factor of 58 (Pirmohamed 2005). Genetic factors may also be related to an ADR; patients with Glucose-6-phosphate dehydrogenase (G6PD) deficiency will suffer from red cell haemolysis when prescribed primaquine.</div>\r\n<div>\r\n	The following steps published by the WHO are usually recommended to be undertaken to identify an ADR (World Health Organization 2002b):</div>\r\n<div>\r\n	&bull; Ensure the medicine ordered is actually taken by the patient at the dose advised</div>\r\n<div>\r\n	&bull; Verify the onset of the suspected ADR was after the drug was taken</div>\r\n<div>\r\n	&bull; Determine the time interval between beginning of drug treatment and onset of adverse event</div>\r\n<div>\r\n	&bull; Evaluate the suspected ADR after discontinuing the drug(s) or reducing the dose. If appropriate, restart the drug treatment to monitor recurrence of adverse event.</div>\r\n<div>\r\n	&bull; Analyse alternative causes</div>\r\n<div>\r\n	&bull; Use up-to-date literature, national pharmacovigilance centre and personal/colleague experience to verify previous reports/experience on the ADR.</div>\r\n<div>\r\n	CAUSALITY ASSESSMENT</div>\r\n<div>\r\n	Once an ADR/E is recognised, the next step is to determine the cause of the ADR/E. Most case reports in pharmacovigilance are for &lsquo;suspected&rsquo; ADR/Es. ADR/Es are rarely specific for a drug, and often without a confirmatory diagnostic test except for a re-challenge which is rarely ethically justified. Few ADR/Es are certain. It is often quoted &ldquo;Report it if you are unsure&rdquo;. The WHO as well Malaysia&rsquo;s national pharmacovigilance centre advises to report an ADR/E even if you are unsure of the cause of the ADR/E or you believe it to be unlikely that an ADR/E has occurred. When in doubt, report it.</div>\r\n<div>\r\n	IMPORTANCE AND RELEVANCE OF IDENTIFYING AND DETECTING ADRS/ADES</div>\r\n<div>\r\n	ADRs affect a significant number of patients. A two year prospective study in the UK involving 19 general practitioners and 872 patients by Martys (1979) reported that up to 41% of patients had some type of adverse drug effect with 90% of the adverse effect occurring by the fourth day of prescription (Martys 1979). A prospective cross sectional study of a representative nationwide (33 hospitals) sample of medical wards by Pouyanne et al. (1998) reported that 3.19% (100 out of 3137) of patients were admitted to hospital because of an adverse drug reaction. The patients tended to be older and were more likely to be female. Four (0.12%) patients died as a direct result of the adverse effect. The authors however do caution that a larger proportion of adverse effects probably occur in the community but do not give rise to hospital admission. A meta-analysis commonly cited as a reference is by Lazarou et al. (1998) which analysed 39 pharmacovigilance studies. The meta-analysis reported the incidence of serious ADRs to be 2.1%, incidence of fatal ADRs to be 0.19% and overall incidence of ADRs to be 10.9% for in hospital patients. The incidence for patients admitted to hospital due to serious ADRs is 4.7% and with fatal ADRs is 0.13%. The overall ADR incidence was reported to be 15.1% of hospital patients. Lazarou et al. (1998) then estimated 106 000 deaths were caused by ADRs in the United States (1994) which would indicate fatal ADRs to be the fourth to sixth leading cause of death.</div>\r\n<div>\r\n	A meta-analysis of 68 studies involving a total of 123 794 patients by Beijer and Blaey (2002) reported that 4.9% (CI 0.1%) of hospital admissions are related to ADRs. Twelve studies provided data on unnecessary hospital admissions, of which 407 out of 1410 (28.9%) were regarded as preventable. Beijer and Blaey (2002) also reported that in people aged over 65 years, the admission rate due to a suspected ADR was higher (16%) compared to the younger individuals (4%). A cost estimate for Netherlands was reported in which the authors estimated that preventable ADR related admissions cost the Netherland health care system was &pound;110 to &pound;256 million, annually ( Beijer &amp; Blaey 2002). A summary of 13 studies by Goettler et al. (1997) reported that ADRs are responsible for a mean length of stay in hospital of 9.6 days with a median of 8.7 days. They also noted that annual direct cost of hospital admissions due to ADRs (1997) for Germany was approximately 1050 Million DM (525 million Euro) and more importantly 30.7% of admissions due to ADRs were preventable, providing a potential savings of 350 million DM (175 million Euro) (Goettler et al. 1997).</div>\r\n<div>\r\n	A two year observational study by Buajordet et al. (2001) examined the cause of death for 732 patients out of a total of 13 992 admissions. Buajordet reported 18.2% (133 out of 732) of deaths were classified as fatal adverse drug events (FADE) with 64 (48%) of the deaths caused directly by one or more drugs. Half of the cases (66/133) were judged to be related to various degrees of inappropriate administration or use of drug(s), therefore potentially avoidable (Pirmohamed et al. 2004).</div>\r\n<div>\r\n	More recent data were provided by Pirmohamed et al. (2004) with his prospective study involving 18 820 patients admitted to two hospitals in Merseyside over a six month period. Pirmohamed et al. (2004) reported that ADRs caused 1225 admissions (6.5%, 95% CI of 6.2% to 6.9%) with 95% classified as type A ADRs. More importantly, Pirmohamed et al. (2004) reported 72% of the ADRs were avoidable. The proportion of women admitted was significantly higher in the ADR group (59% vs 52%) compared to the non-ADR group. 2.3% of patients died as a direct result of the index ADR (0.15% of all patients admitted) suggesting that ADRs are responsible for the death of approximately 5700 patients annually. Patients admitted with an ADR had a median stay of eight days resulting in the projected annual cost of ADR related admissions to the NHS of &pound;466 million. This cost estimate does not include ADRs suffered by patients while being hospitalised and ADRs in primary care that did not result in hospital admissions. A pilot study in 2006 by Davies et al. (2006) assessed ADRs which occurred during hospitalisation rather than the cause of admission. They reported that 19.2% (24 out of 125 patients) of patients suffered one or more ADRs while hospitalised with a median length of stay which was significantly longer than patients admitted for other reasons (14.5 days vs. 8 days) (Davies et al. 2006). With the rising cost of health care (bed occupancy cost and treatment cost), it would be safe to assume that the current cost of ADRs to the health services in the UK should have exceeded &pound;500 million pounds, possibly approaching &pound;2 billion in 2014.</div>\r\n<div>\r\n	ADRs affect all patient populations, although information regarding special populations such as children and pregnant women are limited. In a 10 year retrospective study by Le et al. (2006) at the Miller Children&rsquo;s Hospital, US, the overall incidence of ADRs per hospital admission was 1.6% with an annual incidence of 0.4 -2.3%. However, this study omitted patients with medication errors and patients suffering ADRs out of hospital. Impicciatore et al. (2001) conducted a systematic review and meta-analysis to determine the incidence of ADRs in the paediatric population. The review included 17 studies. Impicciatore reported a higher overall incidence of ADRs among hospitalised children of 9.53% (95% CI: 6.81 to 12.26) when compared with Le et al. (2006) with severe reactions accounting for 12.29% of the total. The overall rate of paediatric admissions due to ADRs was 2.09% (95% CI: 1.02 to 3.77) with 39.3% of the ADRs being severe reactions. The overall incidence of ADRs was 1.46% (95% CI: 0.70 to 3.03) for outpatient children.</div>\r\n<div>\r\n	PHARMACOVIGILANCE IN MALAYSIA</div>\r\n<div>\r\n	It is clear by now that little information has been quoted from Malaysia. Information from Malaysia is scanty and pharmacovigilance researches are frequently underpowered to be referenced. Funding for pharmacovigilance type research is almost non-existent, although clearly lacking and urgently needed. It should be noted that Malaysia has been a member of the WHO Programme for International Drug Monitoring since the year 1990. However, Malaysia&rsquo;s contribution towards the programme could be classified as moderate at best. Although there are currently 118 member countries contributing to the programme, it is widely acknowledged that most contributions to the programme originate from a few countries, mainly the United States, Europe and Japan.</div>\r\n<div>\r\n	Pharmacovigilance in Malaysia is under the responsibility of the Malaysian Adverse Drug Reactions Advisory Committee (MADRAC) which was established under Drug Control Authority (DCA). MADRAC provides DCA with important information pertaining to local and international drug safety issues, as well as advises DCA on risk management and risk communication following effective assessment of the benefit-risk profile of drugs. The National Centre for Adverse Drug Reaction Monitoring acts as the secretariat to MADRAC (MADRAC 2012). The main form of ADR reporting system in Malaysia is a spontaneous reporting system, frequently known as the &lsquo;blue card&rsquo; system as the card used to report ADR is blue in colour. As the name suggests, this form of reporting system is voluntary and is frequently plague by low reporting rates. A systematic review by Hazell and Shakir (2006) reported the median under-reporting rate from 37 studies across 12 countries was 94% with an under-reporting rate of 80% for more serious ADRs. In the UK, the number of ADR reports has remained stable at 25 000 per year for the past 10 years (MHRA 2014). In Malaysia, the number of reports has average at less than 6000 for the past 10 years (Bulletin MADRAC 2014). Reporting of ADR to MADRAC has seen an increase for the past 5 years, doubling from 5850 reports in 2009 to 11473 reports in 2013. This increase is partly due to reinforcement of ADR reporting for immunization, which contributes approximately a fifth of all ADR reports (Bulletin MADRAC 2014). However, further analysis reveals a less positive scenario. Pharmacist contributes the majority of reports (54.7%), followed by ministry of health doctors (13.6%) and product registration holders (10.8%). General practitioners (GPs) and private specialists contributed a meagre 1% of reports. These figures are for the year 2013 (BM 2014) although the trend does not deviate far from this in previous years. Unlike the UK, reports from GPs form the backbone of ADR reporting with total reporting from doctors contributing to almost 50% (MHRA 2014). As doctors, certain important information that would help in causality assessment as well as severity assessment is readily available when compared to other health professionals. MADRAC has now shifted its focus on quality reports instead of quantity. The WHO Uppsala Monitoring Centre measures individual case safety report quality using a score system between 0 (poorly documented case) to 1 (well documented). Over the past five years, reports from Malaysia have consistently obtained an average completeness score of around 0.45 (Bulletin MADRAC 2014). It is however not prudent to advise that only doctors should report an ADR, however the importance and the advantages of doctors reporting ADR encountered must be stressed repeatedly.</div>\r\n<div>\r\n	Spontaneous reporting is the principal pharmacovigilance system in use worldwide with proven effectiveness and a good track record resulting in the avoidance of many potential disasters and the identification of new or previously unknown drug related adverse effects. Its main advantages include wide population coverage, relatively low cost and resource utilisation (given the huge number of population covered and the continuous monitoring of all drugs), and a well established methodology (British Medical Association 2006; Meyboom et al. 1999) However, spontaneous reporting systems depend on voluntary reporting of health care professionals, hence the reporting rate or under-reporting rate becomes the limiting factor determining the success and usefulness of such an approach to pharmacovigilance. In Malaysia, low reporting rates causes signals to be not generated and even if generated, are often late. We instead rely on warnings and precautionary statements from the US and Europe, which is a weakness that needs to be looked into further and not ignored. Drugs marketed in Malaysia, our population, our environment and most importantly our prescribers are inherently different from the US and Europe. Relying on signals from countries with many differences is a weakness which unfortunately is difficult to overcome. The warnings may not affect Malaysia as the drug may not even be marketed here or as an Asian population, we may respond differently. Much more worrying however is the opposite. ADR/Es occurring commonly here but is not detected because no signals is being generated from the US or Europe.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	There is a common misconception especially among the public that all drugs approved by the authorities have all their side-effects or possible adverse reactions known and that the role of the authorities is to monitor any drug on the market for unexpected adverse reactions and to withdraw drugs from the market. In fact, the number of drug withdrawals on the grounds of quality, efficacy, and safety has remained relatively stable since the 70s with only 24 withdrawals out of 583 new active substances (new chemical, biological or pharmaceutical substances for human consumption) approved between 1972 and 1994 with a 10 year survival of all new active substances at approximately 88%, a low attrition rate when compared with other consumer products (Jeffrey et al. 1998). This brings forward an important task for pharmacovigilance, the dissemination of information and educating society regarding drug safety and pharmacovigilance. The message that an approved drug does not mean 100% safety and the identification of unknown adverse reactions after marketing approval is necessary must be presented with tact to avoid mass panic and greater misunderstanding. The recognition of adverse reactions, including serious adverse reactions does not automatically justify the withdrawal of a drug from the market. A balanced assessment between benefit and harm must be made and this assessment should be presented to the public and more importantly to health practitioners, to reduce harm and maximise the benefits. This will however remain a challenge in the foreseeable future for pharmacovigilance and national health authorities in particular, because at the core of this objective coming to realization is improving ADR/E reporting rates.</div>\r\n', 1),
(1231, 1223, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	The lower end of the femur presents two rounded and eccentrically curved condyles. The groove which is present anteriorly between them is called the patello-femoral groove. The notch separating them posteriorly is called as the intercondylar notch (Williams et al. 1995). The intercondylar notch has been an anatomic site of interest as it lodges the anterior cruciate ligament (ACL) (van Eck et al. 2010). The cruciate ligaments have intimate embryological and functional relationship to the intercondylar notch (Miller 2008). The roof of intercondylar notch has an inclination of 40 degrees to the longitudinal axis of femur so that when the knee is in full extension, the roof is near the anterior surface of the ACL (Miller 2008).</div>\r\n<div>\r\n	Palmer (2007) reported for the first time that a narrow intercondylar notch would lead to increased risk of ACL tears. This concept was supported by Souryal et al. (1988) who reported the notch width index as the criteria to estimate risk of ACL injury. It is also reported that the morphology of the intercondylar notch of femur may be clinically relevant in relation to ACL pathologies (Shepstone et al. 2001). There are few articles which have reported about the existence of correlation among the stenosis of femoral intercondylar notch and tears of the ACL (Good et al. 1991; Souryal &amp; Freeman 1993; Davis et al. 1999). The accepted hypothesis is that the size of ACL is related to the size of intercondylar notch of femur (Davis et al. 1999). However there are also some authors who opine that there is no such relation exists (Schickendantz &amp; Weiker 1993; Herzog et al. 1994; Lombardo et al. 2005). Inspite of its clinical interest, there are only a very few study available in the literature about this subject. This was the stimulus to conduct this investigation; the objectives of the present investigation were to study the femoral intercondylar notch morphology and morphometry in cadaver dry bones.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	The present investigation was performed by using 97 dried cadaveric femora (45 belonged to the right side and 52 were left sided). The femora which exhibited damages and pathological changes around the intercondylar notch region were excluded from the present study. The present study has been approved by the ethics committee of our institution. The parameters like intercondylar notch width, intercondylar notch depth, condylar width and condylar depth were measured (Figure 1). The shapes of intercondylar notch were also analyzed. The intercondylar notch was starting at the inferior point of the femoral lateral condyle and continuing medially until the inferior point of the medial condyle of femur. In the present study, the femora were kept on a flat surface of the table and they were resting on their posterior aspects of the condyles and the morphology was macroscopically observed. The shape of intercondylar notches were classified as inverted &lsquo;U&rsquo;shape and inverted &lsquo;V&rsquo; shape.</div>\r\n<div>\r\n	The measurements were performed according to the guidelines of Anderson et al. (1987), Herzog et al. (1994), Wada et al. (1999) and Ravichandran and Melanie (2010). All the measurements of the present study were performed using a digital vernier caliper. The notch width represents the breadth of the notch at about 2/3 part of its depth (Figure 1- CD). The depth of the intercondylar notch was identified as its height at the maximum (Figure 1-AB). The inter epicondylar width was measured as the condylar width (Figure 1-EF) and the maximum antero-posterior height of the lateral femoral condyle was measured (Wada et al. 1999) as the condylar depth (Figure 1-GH). The notch width index and notch depth index were calculated with these data. The width of the intercondylar notch was estimated as the distance between the medial prominence of the lateral condyle to the lateral prominence of the medial condyle. The ratio of femoral intercondylar notch width to the condylar width of femur was determined as the notch width index. The ratio of depth of femoral intercondylar notch to the depth of femoral condyle was determined as the notch depth index. All measurements were were performed by the same person and was observed by the other authors of the present study on each time the measurement was taken. The statistical analysis among the right femora and left femora were performed by the independent t-test, the difference was considered statistically significant if the p-value is less than 0.05. The statistical analysis was performed with the SPSS 15.0 version. The morphometric data of the present study were represented as mean &plusmn; SD. The measurements were compared statistically with respect to right and sides and were tabulated.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	It was observed that the intercondylar notch was having inverted &lsquo;U&rsquo; shape morphology in 71 (73.2%) specimens (Figure 2A) and it was inverted &lsquo;V&rsquo; shaped in 26 (26.8%) cases (Figure 2B). The mean intercondylar notch width, intercondylar notch depth, condylar width and condylar depth were 11.9 &plusmn; 2.7 mm, 26.3 2.4 mm, 72.9 &plusmn; 5.3 mm and 57.3 &plusmn; 4.3 mm, respectively (Table 1). The data were compared on the right and left sides by the independent &lsquo;t&rsquo; test. It was observed that there was no statistical significance difference observed (p &gt; 0.05) between the right and left sides (Table 2). The notch width index and notch depth index were determined as 0.25 and 0.46 respectively.&nbsp;</div>\r\n<div>\r\n	<div>\r\n		DISCUSSION</div>\r\n	<div>\r\n		There are many human and animal studies which reported the correlation between the femoral intercondylar notch width and ACL injury. Since the ACL passes inside the femoral intercondylar notch it is sensible to believe that the morphology of intercondylar notch may influence the function of ACL and the risk of its injury (Shepstone et al. 2001). The morphology of intercondylar notch may be important since the femora with equal intercondylar notch width (or notch width index) can have various shapes. The different shapes of intercondylar notches can give variant quantities of space to the ACL and it is reported that the function of ACL is directly related to the morphology of the intercondylar notch (Shepstone et al. 2001). The space available for the ACL is determined by the dimensions of notch including the depth, width and shape (Ravichandran &amp; Melanie 2010). Notch depth index has been used by some authors to predict the stenosis of intercondylar notch (Wada et al. 1999).</div>\r\n	<div>\r\n		According to an Indian study by Ravichandran and Melanie (2010), the average notch width index was found to be 0.252 and the average notch depth index was found to be 0.467 in cadaver dry bones. Our observations are almost similar to Ravichandran and Melanie (2010) findings with respect to mean intercondylar notch width and depth, condylar width and depth, notch width index and notch depth index. Ravichandran and Melanie (2010) observed inverted &lsquo;U&rsquo; and inverted &lsquo;V&rsquo; shaped notches in 67% and 33% of their specimens.</div>\r\n	<div>\r\n		In the present study, the inverted &lsquo;U&rsquo; shape was found in 73.2% of cases and inverted &lsquo;V&rsquo; shaped in 26.8% cases. These are almost similar to Ravichandran and Melanie (2010) findings.</div>\r\n	<div>\r\n		It was reported that the intercondylar notch was found to be smaller in knees with severe osteoarthritis because of osteophytic growth in the notch (Wada et al. 1999), thus increasing the incidence of ACL ruptures in patients with degenerative arthritis (Leon et al. 2005). Alizadeh &amp; Kiavash (2008) reported that in their study, there was no observed relationship between the narrow femoral intercondylar notch width and the tears of ACL. They also opined that the knee MRI is not recommended to check the chance of ACL tear.</div>\r\n	<div>\r\n		The limitation of this study is the gender based comparison, which has not been done in the present study. The future implications of this study include studying the intercondylar notch by using the radiological techniques like plain X ray of the knee joint, CT scan and nuclear magnetic resonance of the knee joint.</div>\r\n	<div>\r\n		The morphological knowledge of the femoral intercondylar notch may assist the orthopedic and arthroscopic surgeon while inserting the femoral tunnel in the correct location during the ACL reconstruction procedure (Farrow et al. 2007). The morphometry data of the present study could provide of importance to the orthopedicians in the prevention and management of knee injuries. We believe that the present study has provided additional information on this subject and these data might be of use to the clinicians who are involved in the diagnosis and management of knee problems.</div>\r\n</div>\r\n', 1),
(1234, 1226, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Chronic obstructive pulmonary disease (COPD) is a leading growing cause of increasing in morbidity and mortality through out the world. COPD disease represents an increase in burden worldwide, and is reported to be the sixth leading cause of death in the year 1990 and the fourth in the year 2000. Alarmingly, it is expected to be the third leading cause of death by the year 2020. Majority of COPD are at risk of developing cardiovascular complications and about 30% of mortality rate were reported in COPD patients (Lopez et al. 2006). Since, these population has activity limitation because of increased work of breathing, they are prone to develop inadequate exercise intolerance, thereby reducing the cardiac fitness. Numerous rehabilitation strategies and protocols have been employed to reduce the activity limitation in order to preserve their activity or exercise. An earlier study concluded that the heavy resistance training increases muscle size and strength in elderly COPD individuals (Kongsgaard et al. 2004). Similarly, another study found that exercise training was a core component of the pulmonary rehabilitation and it improved the exercise capacity, dyspnoea and health related quality of life (Rochester 2003). In general, it was concluded that circuit training promoted muscle strength, performance in the bicycle ergometry and six minute walk test (Kamahara et al. 2004). An earlier study observed the component of cardiovascular fitness for a period of 6 weeks duration with a predesigned exercise protocol (Borghi-Silva et al. 2009). However, these study results showed different levels of improvements in the fitness level with different protocols and duration. Considering these facts, we designed a specific exercise protocol to know the impact of cardiovascular fitness among the COPD population. Hence, the main objective of the present study was to find out the effect of physical training on blood pressure, heart rate and rate pressure product (RPP) among COPD patients.&nbsp;</div>\r\n<div>\r\n	<div>\r\n		MATERIALS AND METHODS</div>\r\n	<div>\r\n		A total of thirty subjects were selected based on the inclusion and exclusion criteria and were divided in to two groups, experimental (Group 1)-15 patients and control (Group 2)-15 patients who are aged between 40-55 years. Subjects who had COPD and free from other diseases were recruited and randomly assigned. The study was approved and informed consent was obtained from the subjects prior to study. Subjects in both group received routine physiotherapy such as nebulisation and chest physiotherapy where as experimental group additionally received specifically designed physical training for three days a week up to eight weeks. Pulse rate, blood pressure (systolic, diastolic), Rate pressure product (RPP) were recorded as an outcome measure for all the subjects in both group. Pulse rate was measured at the radial pulse and the blood pressure was measured using mercury sphygmomanometer. RPP was calculated as the product of systolic blood pressure and heart rate (Ansari et al. 2012). The mean difference of pulse rate, blood pressure and RPP between the two groups was analyzed using paired t-test.</div>\r\n	<div>\r\n		The experimental subjects were asked to perform specific exercise and each set consisted of 10 repetitions. The duration of each session of therapy lasted for about 30-40 minutes, respectively (warm-up and cool down for 10 min respectively; specific exercise for 20 minutes in standing position) with a rest period lasting for 10 minutes between each sets. The exercise program were as follows: Wrists bend and stretch (flexion and extension), Elbows bend and stretch (flexion and extension), Both arm stretching overhead and reaching towards the floor (flexion), one arm drawn backward and forming a shape of V, alternative foot up and down (ankle raise), one leg then bend and stretched (knee flexion and extension), one leg taken forward and sideways (hip flexion and abduction). The exercise program were altered and monitored based on Rate of perceived exertion Scale &ndash; 10 point -Borg&rsquo;s Scale (Lellamo et al. 2014).</div>\r\n	<div>\r\n		RESULTS</div>\r\n	<div>\r\n		The baseline characteristics showed there was no significant difference in the experimental and control groups before starting the physical training in pulse rate, blood pressure and RPP. The comparison of pre and post test values of pulse rate, blood pressure and RPP for the experimental and control groups were shown in Table 1. Results showed that there was a significant difference between the pretest and post test values of all parameters in Group 1 when compared to Group 2 with p&lt;0.05. The results of the Group 1 were tabulated (Table 2). Diastolic blood pressure showed to be less significant as compared to systolic blood pressure. This showed that specifically designed physical training improved the cardiovascular fitness in COPD patients.</div>\r\n	<div>\r\n		DISCUSSION</div>\r\n	<div>\r\n		The present study results proved that the physical training promoted cardiovascular parameters among COPD subjects who had predetermined protocol compared to those who had routine chest physiotherapy session alone. This present study was supported by a group of authors in which the authors concluded that the endurance training improved cardiovascular fitness in COPD individuals and also causes an improvement in quality of life (Ram et al. 2006). In contrast, a study carried out on the effects of physical training on exercise tolerance showed no significant effect (Mador et al. 2004). The reason behind contrary results could be explained by the facts that physical training alone was carried out in the study where as the present study performed with conventional physiotherapy along with physical training protocol. In one particular study, due to interdisciplinary intervention there was reduced leptin level, in which it indirectly showed improvement in lung function with a duration of one year intervention (Le&atilde;o da Silva et al. 2012). Similarly a study carried out in the year 2003, suggested that exercise training promoted exercise capacity, reduced dyspnea and health related quality of life (Gigliothi et al. 2003). In the present study, the subjects initially showed increase in blood pressure and later on, it got normalized. The possible reason could be the instruction on, how to perform exercise made the subjects nervous and finally increased the blood pressure to a little extent. The physiological reason behind the reduction in the blood pressure was not well understood, but it has been suggested that the reduction in blood pressure were due to the involvement of increased sympathetic activity which decreased baroreflex function and reduced arterial wall compliance. The nitrous oxide produced as a result of aerobic exercise would have caused relaxation of smooth muscles of the blood vessels. We humbly admit few limitations of the study. Initially in the present study most of the subjects involved belonged to the working class. Hence, the activity they got involved also influenced the cardiovascular status. We excluded the Neurology, Cardiac and systemic disease patients. This study was conducted in subjects with in age group of 45 to 55 years which was prone for hypertension. Hence the results obtained could not be generalized to all ages. The study samples were very small and duration is also very less, in future the effect of physical training on respiratory status for COPD should be taken in account.</div>\r\n	<div>\r\n		ACKNOWLEDGMENT</div>\r\n	<div>\r\n		The authors acknowledge the subjects who participated in the study.</div>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1241, 1233, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Medical school programme for undergraduate is divided into two phases: pre-clinical and clinical. The clinical phase is when they will visit the clinics, wards and even patients&rsquo; home in order to get contact with the patients. The study techniques, ward rounds, tutorial, lectures and seminars are are very demanding that can lead to psychological distress among the students (Zaid et al. 2007). Psychological distress is to define as emotional instability which includes studentstress, anxiety and depression (Drapeau et al. 2012).</div>\r\n<div>\r\n	Anxious students may have palpitation, difficulty in breathing, numbness and gastrointestinal symptoms which can affect their attention and study (Desrosiers &amp; Miller 2007). More than half of our university students (non-medical and also medical) were found to suffer from anxiety while studying the undergraduate course (Shamsuddin et al. 2013; Yusoff et al. 2013). Students become anxious especially when they are worried about failing an exam. Students who are anxious may easily become distressed with their anxiety symptoms.</div>\r\n<div>\r\n	Stress is very common among medical students. It is described as a fearful anticipation accompanied by sweating and increased heart beating (Benjamin &amp; Virginia 2005). Shah et al. (2010) stated that the stressors are parental expectations, frequent examinations, the enormous academic curriculum, worry about their future and academic performance. Females are found to be more stressed than males. It is possibly due to their personalities and different coping mechanism. An earlier study established that most students perceived examination as the main stressor (Shah et al. 2010). The academic stressors may lead to psychological stress and subsequently depression if they do not have good stress management.</div>\r\n<div>\r\n	Depression is a psychopathological mood occured for more than two weeks disturbing the student&rsquo;s work and social function (Benjamin &amp; Virginia 2005). About 15% of medical students was found to be depressed.Since feeling depressed was associated with increased risk of suicide, it is not surprised that one fifth of them had suicidal ideation (Fan et al. 2012; Tija et al. 2005).</div>\r\n<div>\r\n	Medical course is well known with its stressful pursuit that often causes a negative impacts on physical and mental health of the students. Students might not be able to face all the stressors in their medical life; hence, they should seek help from the right sources. The sources of help can be divided into formal (counsellors, lecturers, health professionals, social workers, religious personnel or youth programmes) and informal (friends, family members and peer groups) (Tuisku et al. 2006).</div>\r\n<div>\r\n	Hence, the present study was performed to identify psychological disorders among our medical students who were in their clinical years. Their help seeking behavior was also explored.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a cross-sectional study conducted in UKM Medical Centre, Malaysia. Participants in this study were in their third to fifth year of medical school. By using proportional sampling method, students from each academic year were selected randomly. The students were approached individually (before or after the formal teaching, or in their dormitories). The students were briefed about the study and they were provided with a set of self-administered questionnaire and a consent form.This questionnaire consisted socio-demographic items, BM DASS-21 and questions on help-seeking behaviour.</div>\r\n<div>\r\n	The validated BM DASS-21 comprised of 21 questions that gauge the severity level of depression, anxiety and stress. However, it is not a diagnostic tool. The internal consistencies for each domains of BM DASS-21 were very good: depression (0.84), anxiety (0.74) and stress (0.79) (Ramli et al. 2007).</div>\r\n<div>\r\n	The questions on help-seeking behaviour assessed the use of medical facilities and other help-seeking sources. It was adapted from previous study by Aida et al. (2010). Face validity was done to improve its clarity and comprehensibility.</div>\r\n<div>\r\n	The collected data was analyse dusing the Statistical Package for Social Sciences version 18.0 (SPSS 18.0). Descriptive analysis in frequency and percentage were used to present the data of socio-demographic and BM DASS-21. Pearson&rsquo;s Chi-Square with Yates Correction was used to analyse the association between the students&rsquo; psychological disorders and their help-seeking behavior. The statistical significance was established when p value of &lt;0.05.</div>\r\n<div>\r\n	Ethical approval from the Research Committee of Universiti Kebangsaan Malaysia was obtained prior to the study. Written consent was also obtained from the students prior to their participation.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	There were 458 medical students approached but only 380 students agreed to participate in this study (82.9% response rate). There were 221 females (58.2%) and 159 males (41.8%) with their age ranged between 20 and 25 years old (SD 1.049). Majority of the students were Malays (243; 63.2%), followed by Chinese (132; 34.7%), Indian (5;1.3%) and others (3; 0.8%). The respondents were third year (147; 38.7%), fourth year (118; 31.1%) and fifth year (115;30.3%) medical students. Majority of them had good Cumulative Grade Point Average (313; 82.4%). More than half of the respondents stayed in dormitories (311; 81.8%) and were active in co-curriculum activities (190;50%). Majority of the students stated that they had good relationship with their parents (374; 98.4%) (Table 1).</div>\r\n<div>\r\n	It was found that only minority of the students had depression (1.3%), anxiety (2.4%) and stress (2.4%) (Table 2). Majority of them preferred to seek help from friends (283; 74.5%) followed by parents (275; 72.4%) and siblings (203; 53.4%) (Table 2). Among formal sources of help, religious personnel (71; 18.7%), psychiatrists (64; 16.8%) counselors (45; 11.8%) were the top three preferred sources by the students (Table 3).</div>\r\n<div>\r\n	When we analysed the associations between students psychological disorders (depression, anxiety and stress) and their help seeking behaviour (Table 4 and 5), we found that there was a significant association between the presence of anxiety and help-seeking behaviour from the formal sources: counsellor (p=0.001) and religious personnel (p=0.03) (Table 5). However, there was no significant associations between the other psychological disorders (depression and stress) and other sources of help (Table 4 and 5).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This cross-sectional study had a good response rate, particularly among the third year medical students. Moderate response rate from the fourth and fifth year students might be due to their heavier clinical work as compared to the third year students. This is similar to a study by Abdul Ghani (2008). Most of the respondents in this study were Malay (63.2%), followed by Chinese (34.7%) and Indian (1.3%). This trend is similar to that of the national ethnicity trend in which Malays is the biggest ethnic, followed by Chinese and Indian.</div>\r\n<div>\r\n	An interesting finding in this study was a low prevalence of psychological disorders among the UKM medical students (depression: 1.3%, anxiety: 2.4% and stress: 2.4%). The prevalence was lower compared to a previous study done in Northern Europe where 14% of their medical students were depressed and 43% of them were anxious (Bunevicius et al. 2008). This could be due to post-exam sampling of the students in our study. The UKM medical students could also have good strategies to cope with their emotional problems. Another possible reason is implementation of mentoring programme during the clinical years. This programme involves assigning four to five students to one lecturer who acts as a mentor. They have scheduled meetings to discuss about their academic performance and any psychosocial issues with their mentors.</div>\r\n<div>\r\n	There are two types of help seeking behaviour, formal and informal (Tuisku et al. 2006). In this study, medical students preferred informal sources of help such as friends (74.5%), parents (72.4%) and siblings (53.4%) compared to formal sources of help. This finding was similar to another local study that involved adolescents from a secondary school (Aida et al. 2010). This could be due to their good relationships with their friends (98.7%), parents (98.4%) and siblings (98.7%). Other studies also reported that many medical students shared their problems with their friends as their coping mechanism (Sherina et al. 2003; Zaid et al. 2007).</div>\r\n<div>\r\n	Staying together with friends, sharing similar academic stressors and having good friendships could explain their preference to seek each others&rsquo; help to deal with any problems including emotional disturbances. In addition, parents are found to be an important source of support particularly when their emotional distress arises from financial problems (Sherina et al. 2003).</div>\r\n<div>\r\n	Very few of them trusted people they met in the internet chat rooms to share their problems. However, Gould et al. (2002) reported a higher proportion of adolescents sought support from people in the internet. The low use of internet chat rooms as a source of help in our study could be because they were well connected with their friends through social media such as Facebook, WhatsApp and Twitters. The medical students should also be informed about the reliable websites, for instance Caf&eacute;@Teen by Natural Population and Family Development Board Malaysia that provide help in managing their emotional problems (LPPKN 2008).</div>\r\n<div>\r\n	With regard to the formal sources of help, majority of the students did not seek support from the psychiatrists, counsellors, religious personnel and other healthcare providers. They might be afraid that these healthcare providers could have negative perception towards them. Since there is a possibility that they will be working with these healthcare providers, the negative perceptions could jeopardise their future relationships with them. In addition, they also might have doubt that their confidentiality would be breached. Furthermore, meetings the healthcare providers for help could cause them to miss their classes, lectures and clinical teaching sessions. Thus, their performance might be affected and could make them more distress.</div>\r\n<div>\r\n	Interestingly, there was a significant association between the presence of anxiety and help seeking behaviour from the formal sources. Significantly more students who had anxiety sought help from the counsellors and religious personnel compared to those who were not anxious. These results were similar to an earlier study done by Eisenberg et al. (2007). There was no significant association between emotional distress and help that these students sought from informal sources. Rosenthal and Okie (2005) found that many students perceived stress in medical school was common. Hence, there was no need for them to seek help from others.</div>\r\n<div>\r\n	Limitations of the study were related to the cross-sectional design and the timing of data collection which was done following an examination. Students might have less emotional distress during this period. Therefore, we recommend future studies to be performed prospectively.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Prevalence of psychological disorders among UKM medical students was low. Significantly more students who were anxious sought formal help from counsellors and religious personnel. Good relationships with friends and parents were essential for their psychological well beings. Eventhough the prevalence was low, the students&rsquo; awareness on these disorders cannot be determined. An effective screening programme is still needed to recognise students with possible psychological disorders.</div>\r\n<div>\r\n	ACKNOWLEDGEMENTS</div>\r\n<div>\r\n	I would like to thank Universiti Kebangsaan Malaysia for funding this research. I would also like to express gratitude to Professor Dr Ramli Musa for sharing the validated BM DASS 21.</div>\r\n', 1),
(1248, 1240, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Early identification and treatment of sepsis are essential to improve the outcome of sepsis patient or patient with impending sepsis (Rivers et al. 2001; Shapiro et al. 2005). Static lactate elevation had been utilized as a key to predict mortality rate of sepsis patients in emergency department (ED) (Bakker et al. 1991; Aduen et al. 1994; Bakker et al. 1996; Bakker 2001; Dellinger et al. 2004a; Varpula et al. 2005; Marty et al. 2013). Current Surviving Sepsis Campaign (Dellinger et al. 2004b; Nguyen et al. 2004) advocates measurement of lactate in sepsis patient and possible severe sepsis patient to guide the further management (Shapiro et al. 2005). On the other hand, an&nbsp;early lactate clearance had also shown improvement in the outcome of sepsis as well (Fuller &amp; Dellinger 2012; Walker et al. 2013) and few other studies had shown its prognostic outcome (Varpula et al. 2005; Fuller &amp; Dellinger 2012; Puskarich et al. 2012). Furthermore, lactate clearance has been proposed as a better therapeutic guidance for severe sepsis and septic shock patient&rsquo;s management (Kruse 1999; Shapiro et al. 2005; Wang et al. 2006; Jones et al. 2010; Jansen et al. 2010; Jones 2011). Lactate clearance also suggested being less invasive and more cost effective in treating sepsis (Varpula et al. 2005). However, some studies had point out the flaw of static lactate and lactate clearance as risk stratification. Many sepsis patients are presented with normal static lactate (Mizock 2001; Dugas et al. 2009; Hernandez et al. 2011). Static lactate level is inconsistent with fluctuation after intervention (Hernandez et al. 2011, Rivers et al. 2011). However, a research has found out that lactate clearance does not improve the outcome in septic syndrome patients (Jones et al. 2010). Another study also proposed that lactate clearance should not be used as a substitute guideline for management severe sepsis and septic shock with reasons that inconsistency of static lactate is inappropriate risk stratification and lactate clearance can be a delayed indicator of tissue perfusion with lack of understanding on mechanism behind it (Marty et al. 2013). In addition, better lactate clearance does not necessary improved outcome of septic syndrome patient (Marik &amp; Bellomo 2013). These arguments had triggered us to elucidate (a) the association of static lactate and lactate clearance at 0 hour, 1 hour and 6 hour, respectively with 28-day mortality in sepsis syndrome of the ED and (b) to examinine for 28-day mortality prognostication of static lactate level and first six hours lactate clearance during admission in ED for sepsis syndromes.</div>\r\n<div>\r\n	MATERIALS AND METHODSSETTING AND STUDY DESIGN</div>\r\n<div>\r\n	This was a prospective observation study which was performed in ED of Universiti Kebangsaan Malaysia Medical Centre (UKMMC) from March 2014 to May 2014. The study population consisted of severe sepsis, septic shock and sepsis patients who were admitted in ED during the study. The initial venous or arterial blood serum lactate level at 0 hour, 1st hour and 6th hour were measured. After 28 days, a follow-up was pursued in each patient to determine all-cause mortality.</div>\r\n<div>\r\n	PARTICIPANTS</div>\r\n<div>\r\n	The inclusion criteria were consented patients, suspected sepsis patients who older than 17-year-old with two or more criteria of systemic inflammatory response syndrome. Besides that, patients with systolic blood pressure (SBP) less than 90 mmHg after 500 to 1000 ml crystalloid fluid challenge also recruited in our study. However, the exclusion criteria are the patients who were partially treated with antibiotics for more than three days, or patient deceased during the period of recruitment. Patient transferred out to other hospitals without completing the six hours management were also excluded from this study.</div>\r\n<div>\r\n	VARIABLES</div>\r\n<div>\r\n	Sepsis is defined as infection with two or more criteria of Systemic Inflammatory Response Syndrome (SIRS). In addition, definition of severe sepsis is sepsis associated with at least one tissue hypo-perfusion or present of sepsis-induced organ dysfunction. Furthermore, septic shock which is also known as hypotension induce by sepsis is defined with a mean arterial pressure &lt; 70 mm Hg or a SBP decrease more than 40 mm Hg or &lt; 2 SD below normal, or systolic blood pressure (SBP) less than 90 mm Hg for age, which there is no other causes responsible for the hypotension despite adequate fluid resuscitation. Moreover, the definition of sepsis induced tissue hypo-perfusion is oliguria, septic shock, or elevated lactate (Table 1). Lactate clearance is defined as blood lactates reduced by 10% or more from the initial blood lactate levels. It can be calculated by using the formula as follows:</div>\r\n<div>\r\n	[(LACinitial &ndash;LACfinal)/ LACinitial] x 100%]</div>\r\n<div>\r\n	STUDY SOURCES/ MEASUREMENT</div>\r\n<div>\r\n	Once the patient has fulfilled the inclusion and exclusion criteria, blood lactate was withdrawn (2 ml) at H0, H1 and H6. The samples were collected into sodium fluoride/ potassium oxalate (grey top) test tube. After that, the blood was centrifuged (1300G, 10 minutes) and sent for lactate analysis. A standard treatment of sepsis was initiated at 0 hour for patients who were identified as sepsis. For sepsis-induced hypo-perfusion patients, they were treated using Early Goal Directed Therapy (EGDT). After 28 days, a follow-up report was conducted via email, through case notes, or telephone call. Following that, patients were sort into two categories which are non-survivor or survivor as an outcome.</div>\r\n<div>\r\n	STUDY SIZE</div>\r\n<div>\r\n	Power and sample size was calculated using Open Epi Version 3 based on Fleiss Statistic Method of Rate and Proportion. Calculation of sample size was based on preliminary serum lactate result of our local study, which the sensitivity is 75% and specificity is 77%. For two-sided significant level, the percentage of general population included = 95%. Power of the study = 80%. Odds ratio, the possibility to get disease if being exposed as compare to normal people = 4.9. Ratio of sample size exposed or unexposed = 1. Using Open Epi Version 3, by Fleiss Statistic Method of Rate and Proportion, we estimated that a minimum of 144 total cases would be necessary.</div>\r\n<div>\r\n	STATISTICAL METHOD</div>\r\n<div>\r\n	Statistical analyses were performed using SPSS version 20.0. The mean &plusmn;standard deviation represents quantitative variables while number and percentage represent qualitative data. Kolmogorov-Smirnov test was used to test normal distribution of data. Fisher&rsquo;s exact test was used in categorical variables while Mann-Whitney U was used in continuous variables test for comparison of survivors and non-survivors. All tests used were two-sided, and statistical significance was determined with p&lt;0.05. Receiver-operating characteristics (ROC) curves and area under the curve (AUC) were computed.</div>\r\n<div>\r\n	RESULTSCHARACTERISTIC OF STUDY COHORT</div>\r\n<div>\r\n	From March to April 2014, a total of 375 patients admitted to Emergency Department were suspected with sepsis syndrome. From this amount, 40 patients were eligible and recruited for this study. Twenty one patients were excluded (15 not consented, 6 incomplete data collection). Total of 19 patients data were analyzed. Demographic data are provided in Table 2.</div>\r\n<div>\r\n	STATIC LACTATE AND LACTATE CLEARANCE</div>\r\n<div>\r\n	The overall mortality for both group were 25% in lactate non clearance group versus 75% in lactate clearance group. The median static lactate for survivor and the non-survivor at H0, H1 and H6 are showed in Table 3. The results for lactate clearance percentage for both groups at H0-H1, H1-H6 and H0-H6 are showed in Table 4. We found that the threshold for static lactate level to predict 28-day mortality in sepsis for H0, H1 and H6 were 3.9mmol/L, 1.9mmol/L and 2.3mmol/L respectively (Table 5). The ROC curves for that lactate clearance are showed in Table 5. DISCUSSIONSTATIC LACTATE</div>\r\n<div>\r\n	All static lactate threshold level was significantly lower in the survivor group from the non-survivor at H0, H1 and H6. The median static lactate for survivor is significantly lower than the non-survivor at H0, H1 and H6 (p&lt;0.05) (Table 3). Based on ROC curves, the AUR for at H0, H1 and H6 suggested that static lactate as a good prognostic tool to predict in 28 days mortality for sepsis. The difference of static lactate between survivors and non-survivors suggests that higher static lactate associates with the increment of mortality rate. Initial static lactate level does correlate with mortality rate thus provides a good prognosis for sepsis (3.85 mmol/L).</div>\r\n<div>\r\n	LACTATE CLEARANCE &ndash; A TOOL FOR MONITORING SEPSIS?</div>\r\n<div>\r\n	In our study, despite there is a lactate reduction trend at H6, lactate clearance showed a weak association in predicting 28-day mortality in sepsis patients which supports similar findings by Marty et al. (2013). The most plausible explanations for lactate clearance are the increased lactate removal, decreased lactate production, and dilatation effect due to fluid resuscitation (Jones 2011). Many studies showed that the rise of lactate production after initial resuscitation effort could be due to activation of glycolytic pathway triggered by mechanism such as Na+/K+-ATPase rather than the impact of tissue hypoxia (Hernandez et al. 2010, Gutierrez &amp; Wulf 1996, James et al. 1999, Hotchkiss &amp; Karl 1992, O&rsquo;Brien et al. 1996). It is also suggested that high initial lactate levels had caused irreversible organ damage (Jansen et al. 2009). Thus, aggressive resuscitations are doubtful to reduce the impact of tissue damage caused initial high static lactate. Besides that, lactate is also a delayed indicator of tissue perfusion which the mechanism is yet to be elucidated (Marty et al. 2013). Age is a possible confounder for static lactate predicting 28-day mortality. Further regression analysis should be carried out in the larger sample size to elucidate this trend.</div>\r\n<div>\r\n	This study supports recommendation by survival sepsis campaign, which suggests the EGDT should be initiated on patients with static lactate more than 4.0 mmol/L (Hernandez et al. 2010). Interestingly, static lactate at H1 has a threshold 1.9 mmol/L, and it appears to be lower compared with H0 and H6. It may have two possible explanations. First, it may reflect the successful of aggressive resuscitation. Secondly, the dilution effect of fluid resuscitation may lower the lactate level. This marked depression of lactate is not preserved throughout the treatment period. This reflected the resuscitation effort may have slowed down after patient was more stable after the achieving a lower static lactate level at H1.</div>\r\n<div>\r\n	LIMITATION OF STUDY</div>\r\n<div>\r\n	There were few limitations to be considered in this study. First of all, this study was done in one hospital, this might have influenced the results. Our study may introduce a selection bias that was difficult to account for. We were unable to keep track and take the blood samples on time as the acutely ill patient was highly mobile in ED to different zone from time to time according to their dynamic of the illness during the recruitment. We were also facing difficulty in getting the consent from the acutely ill patient in the absent of the next of kin. We tried to include as many patients as possible who have fulfilled the sepsis criteria to achieve the estimated sample size (n = 144). However, targeted sample size was partially achieved. The accrual time to reach 144 patients was estimated to be 24 months. It is possible that a larger study may result in different conclusion. The age profile cannot be met as demographic data shows non-survivors appeared in older age group. It prevents further modeling to be performed. Information bias is possible because some information may not have been properly recorded.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Our data has shown that with limited number of samples, static lactate was found to be useful to determine survival of sepsis patients. In our centre, static lactate remained as a strong dependent predictor for sepsis in hospital death. In our study, static lactate is suggested to be superior over lactate clearance as a predictor for sepsis in 28 days mortality. However, lactate clearance can be a useful tool to monitor the resuscitation effort.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	We thank the doctors, assistance medical officer, nurses, technical and administrative support staff in the Department of Emergency Medicine of Universiti Kebangsaan Malaysia for their assistance in this study.&nbsp;</div>\r\n', 1),
(1252, 1244, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Although, the management of premature infants has improved over the years, the long-term morbidity of these infants, especially due to conditions such as retinopathy of prematurity (ROP), is still a great challenge. The risk factors for developing ROP are birth weight less than 1500gm and prematurity of less than 32 weeks (Palmer et al. 1991). ROP accounts for 3% of all cases of childhood blindness (Rahi &amp; Cable 2003). ROP occurs as a result of hypoxia of the eye due to incomplete development of the retinal vessels. The severity of the disease is determined on the basis of the size of the avascular area, amount of abnormal vessels, and the presence of the features of plus disease that increase the risk of further disease progression. Treatment was recommended only if threshold criteria were met (Palmer et al. 1991).</div>\r\n<div>\r\n	The incidence of blindness from untreated ROP has been significantly reduced by the availability of treatment (Hardy et al. 1997). Management of peripheral retinal ablation either with laser treatment or cryotherapy induces rapid regression of abnormal vessels and allows for vascularization to continue normally towards the ora serata. According to the findings of the Cryotherapy for Retinopathy of Prematurity (CRYO-ROP) study, treatment is necessary only for eyes with &ldquo;threshold&rdquo; disease. Even if successful regression of the disease is immediately after treatment, other ocular morbidities ensue with the growth of the infant. Strabismus and amblyopia are still being reported as the long-term morbidities observed in such cases (VanderVeen et al. 2011).</div>\r\n<div>\r\n	Immaturity of the retina is described on the basis of the area of the avascular zone; presence of changes in zone 1 is associated with the most severe form of immature retina, while those in zone 3 were the least severe form of the incomplete development of the retina (Cryotherapy for Retinopathy of Prematurity Cooperative Group 1988). Plus disease is defined by the presence of arterial tortuosity and venous dilatation, which are the critical factors for evaluating treatment. While the threshold for treatment is at least five contiguous or eight cumulative sectors (clock hours) showing stage 3 ROP in zone 1 or II in the presence of plus disease (Cryotherapy for Retinopathy of Prematurity Cooperative Group 1988).</div>\r\n<div>\r\n	Although ROP is commonly reported as a bilateral disease, a small percentage of infants have asymmetrical changes. Hence, the indication of treatment may differ and one eye might remain untreated. To the best of our knowledge, no detailed report has been published on the long-term outcome of unilateral ROP treatment.We present a case demonstrating one of the possible long-term outcome in a patient with peripheral retinal ablation in a single eye for threshold ROP. At the time of treatment constitution, the CRYO-ROP study was the main treatment guideline.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	The patient was a baby girl born at 29 weeks of gestation in 2004 with a birth weight of 1500 gm. At birth, the patient had respiratory distress syndrome and received respiratory support for more than three weeks. Her general condition improved with the treatment. During routine ophthalmologic screening, bilateral changes were consistent with ROP detected at 34 weeks following conception. There were asymmetrical changes in both eyes. The patient&rsquo;s left eye fundus revealed changes of &ldquo;threshold&rdquo; ROP (stage 3 ROP with changes for more than 5 clock hours and preretinal hemorrhage in zone 2 with plus disease). However, the right eye showed less severe changes (stage 2 ROP changes less than 4 clock hours in zone 2) with no evidence of plus disease. As per the CRYO-ROP criteria, treatment was indicated only in left eye that had threshold disease. Therefore, indirect argon laser treatment was administered only for the left eye. Meanwhile, no treatment was administered to the contralateral eye with less severe disease and not meeting the threshold criteria.</div>\r\n<div>\r\n	The infant responded well to the treatment, and both eyes showed rapid regression of the abnormal vessels. The patient was examined every six months and evaluated for refractive errors every six months. At six months of age, the spherical equivalent was found to be +0.75 D in both eyes with no evidence of squint at the beginning. The patient&rsquo;s myopic changes progressed more rapidly in the left eye than in the right eye. Myopic changes at 18 months of age showed significant differences between the two eyes. The spherical equivalent of the treated eye was -2.25 D, while that of the untreated eye was only -0.30D. Significant difference in refractive error between the two eyes putting her at risk to develop amblyopia in the future. Patching of the good eye which is one of the treatment modalities to prevent amblyopia was instituted early. However, the patient did not comply with this treatment. Anisometropia (significant different of the refractive error between the two eyes) progressed with her age. At eight years of age, the myopia in her left eye worsen to -6.25D with best-corrected visual acuity of 6/36, while her right eye remained emmetropic (no significant refractive error) with 6/6 vision. Ocular biometry at the age of 8 years revealed steeper corneal curvature in the left eye,with a longer axial length of 24.17mm in the affected eye and 21.37mm in the other eye. With the resultant anisometropia and poor compliance to treatment, the patient experienced dense amblyopia with subsequent squint of the left eye.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	In premature infants, prolonged exposure to assisted ventilation, which may be necessary for survival, is often detrimental to the eye, leading to conditions such as ROP (Todd et al. 1990). ROP is a disease affecting the development of the immature vasculature of the eyes of premature babies. It presents in various grades of severity, ranging from very mild disease without any significant visual effect or very aggressive disease with abnormal vessel proliferation resulting in retinal detachment and blindness. Asymmetrical presentation of ROP is not uncommon; it has been reported that the differences in the severity of the eyes of the same patient with only one eye requiring treatment occurs in up to 20% of cases (Azad et al. 2010).</div>\r\n<div>\r\n	The infant in this case developed threshold changes with plus disease in the left eye and less severe form of ROP without the presence of threshold in the fellow eye. Hence, treatment was constituted only in the eye meeting the threshold criteria in accordance with the standard guideline of CRYO-ROP, which was the main treatment guideline at the time of presentation. Peripheral retina laser ablation is the main stay of treatment.</div>\r\n<div>\r\n	Despite reducing the risk of complete blindness, significant refractive error particularly myopia (short sighted) which may lead to amblyopia is still frequently reported in a high proportion of treated infants (Al-Gamdhi et al. 2004). Amblyopia (also known as lazy eye) is a condition which involves reduced vision in an eye that otherwise appears normal, or out of proportion to associated anatomical problems of the eye. Detecting the condition in early childhood increases the chance of successful treatment.</div>\r\n<div>\r\n	In this particular case, at the age of eight years, the infant was found to have a longer axial length in the treated eye than in the other eye which may explain the significant refractive error condition. The mechanisms of myopia in infants with ROP are debatable. Most experts agree that the myopic effect is the result of poor anterior segment development in ROP, particularly of the lens and cornea (Garcia-Valenzuela &amp; Kaufman 2005). At the same time, the incidence of myopia in treated infants was reported to correlate with increases in the stage of ROP, clock hours of involvement, number of laser spots, and period of regression (Katoch et al. 2011).</div>\r\n<div>\r\n	However, others have also reported that the possible effect of treatment particularly peripheral retinal ablation may interfere with the normal ocular development (Ng et al. 2002). Therefore, the exact mechanism of myopia in ROP with or without treatment is still poorly understood.It is always presumed that regression of ROP and resumption of the normal development of retinal vessels will help the patient achieve good visual acuity in the future. However, obviously regression of the disease is not the only determinant factor for a good visual outcome.</div>\r\n<div>\r\n	It is important to highlight the possibility of unequal development of the eye in asymmetrical presentation of ROP. The unresolved issues regarding the long-term outcome of ocular development in ROP patients warrants consideration during the planning of treatment in asymmetrical cases. Strict monitoring of amblyopia treatment would help to prevent poor visual outcome in such cases.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Asymmetrical presentation of ROP and the resultant differences in the&nbsp;treatment regimens for both eyes may have different effects on the long-term ocular development of the eyes of the patient. Although regression of ROP after treatment increases the chances of achieving a useful level of vision in the patient, regular monitoring and strict intervention are equally important for the management of significant refractive error to avoid poor vision in the future.</div>\r\n', 1),
(1255, 1247, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Schizophrenia is a multifaceted chronic disabling illness which poses a significant cost at the distress of the patient and to the burden of the caregiver. The traditional hospital-only psychiatric services have been shown to be inadequate in providing comprehensive care for people with mental illness (Coldwell &amp; Bender 2007). This case highlights the burden faced by a family with three members suffering from schizophrenia and the role of ACT in improving their clinical condition and relieving their suffering.</div>\r\n<div>\r\n	CASE REPORTMADAM A</div>\r\n<div>\r\n	Madam A was a 48-year-old lady, widowed since 20 years ago and had two children out of consanguineous marriage. She was diagnosed with schizophrenia at the age of 28 years where she presented with second and third person auditory hallucinations and delusions of persecution and reference. Soon after the diagnosis, she developed marked depressive symptoms with active suicidal ideation secondary to the frightening psychotic experiences.</div>\r\n<div>\r\n	Since the first episode, she had never attained full remission. She had frequent relapses requiring hospitalizations partly due to her poor insight leading to non-adherence to medications and at times triggered by stressors present in the family. She was treated with a series of antipsychotics including haloperidol, chlorpromazine and, risperidone with suboptimal response. Her mother, Madam D, claimed to supervise her medications. However, it could not be ascertained as she was frequently distressed having to care for Madam A and two other sick family members. Madam A was once tried with clozapine with total dose of 700 mg daily which yielded better clinical response. However, due to her erratic intake of this oral medication, in addition to the risks of severe side effects the medication may pose, it was switched to intramuscular depot flupentixol and tablet olanzapine which resulted in similar satisfactory response. Unfortunately, olanzapine was later substituted with the cheaper haloperidol due to financial reason after her Parkinson-stricken father passed away with the termination of financial benefit which he used to receive from the Welfare Department.</div>\r\n<div>\r\n	Electro-convulsive treatment (ECT) was once commenced during one hospitalization and was continued on maintenance basis. However, she defaulted mainly due to the difficulty that the mother had to bring her to the hospital.</div>\r\n<div>\r\n	She was referred to the Community Mental Health Team (CMHT) of Universiti Kebangsaan Malaysia Medical Center (UKMMC) since last seven years. Through regular home visits of weekly to monthly by the team, her medications were monitored more closely and the family was helped to overcome the financial problem by engaging the relevant agencies. These services helped to gradually reduce her hospital admissions from four times a year to an average of once a year.</div>\r\n<div>\r\n	<div>\r\n		MISS B</div>\r\n	<div>\r\n		Miss B, the younger sister of Madam A; a 44-year-old single lady, first presented to UKMMC at the age of 22 years and was diagnosed with mental retardation with schizophrenia. She had history of multiple psychiatric admissions with typical presentation of florid auditory hallucinations and disorganized behaviour, causing significant distress to the family. She also received similar services from the CHMT which helped to stabilise her condition. Her last admission was four years ago. Her symptoms were controlled by sulpiride 200 mg bid and IM fluphenazine 25 mg, monthly.</div>\r\n	<div>\r\n		MISS C</div>\r\n	<div>\r\n		Miss C, the eldest daughter of Madam A, currently 24-years-old, was diagnosed with mental retardation since younger days. She was first hospitalized when she was 19 years due to psychosis and disorganized behavior which disrupted the family&rsquo;s function, as she needed extra care. She subsequently required frequent admissions two to three times a year when she relapsed with similar presentations. She was diagnosed with mental retardation and schizophrenia. Her symptoms were well controlled when she was on olanzapine. However, due to financial constraint; olanzapine was substituted with sulpiride at one stage, which had destabilized her illness with impending relapse. The CMHT managed to secure financial aid to sustain her previous medication. Currently she is doing well on olanzapine 15 mg nocte. With regular supervision of her medication by the CMHT, she had maintained to be symptom free and had not required hospitalization for a long time. She also regularly participated in group psychosocial rehabilitation activities scheduled by the team which provided the family some respite from caring for her.</div>\r\n	<div>\r\n		MADAM D</div>\r\n	<div>\r\n		She was a 68-year-old lady and a widow, who out of consanguineous marriage was blessed with two daughters; Madam A and Miss B. She was the only mentally healthy care-taker in the family; who had to care for her mentally ill daughters, grand-daughter and husband who suffered from Parkinson disease for many years before his death. She developed major depressive disorder seven years ago with family wellbeing and financial issues being her constant worries. She also suffered from long-standing diabetes mellitus, hypertension and had a right lacunar infarct four years back. She had a few admissions to the Medical Ward, due to the complications of her medical illness but had never been admitted to the Psychiatric Ward.</div>\r\n	<div>\r\n		Treatment with escitalopram 15mg nocte helped to control her symptoms. However, she would still have bouts of low mood when faced with difficulties in caring for her mentally ill family members. In view of her family&rsquo;s condition, she felt stigmatized and isolated from the community. She took care of the household and managed her mentally ill family members. The CHMT had worked with her in creating medication charts to improve medication adherence. The team was also vigilant in monitoring her mood and giving her emotional and practical support. This involved mobilizing support from relatives and relevant agencies.</div>\r\n	<div>\r\n		DISCUSSION</div>\r\n	<div>\r\n		This report demonstrated the complexity of managing a family with its households suffering from mental illness and what ACT could provide in restoring remission and improving their quality of life. ACT which is the earliest available component of community-oriented services in Malaysia, functions to integrate and organize psychiatric care for patients with severe mental illness to cater for their different needs at different stage of life (Ruzanna &amp; Marhani 2008). As demonstrated in this case, ACT has been proven in one study to reduce hospitalization, retain contact with service, improve patient satisfaction, and reduction of illness severity (Lambert et al. 2010). In accordance with the latter finding, a local study showed a high prevalence of symptomatic remission among schizophrenia patients who received ACT (n: 118, remission rate 78%) (Dahlan et al. 2013). In keeping view with all these facts, ACT should be provided to patients who are at high risk of repeated hospitalization and homelessness (Dixon et al. 2010).</div>\r\n	<div>\r\n		In the present case, ACT helped one entire family in their journey of living with mental illness. ACT could be an essential psychiatric service component that should be established at all localities.</div>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1258, 1250, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Schizophrenia is a multifaceted chronic disabling illness which poses a significant cost at the distress of the patient and to the burden of the caregiver. The traditional hospital-only psychiatric services have been shown to be inadequate in providing comprehensive care for people with mental illness (Coldwell &amp; Bender 2007). This case highlights the burden faced by a family with three members suffering from schizophrenia and the role of ACT in improving their clinical condition and relieving their suffering.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	MADAM A</div>\r\n<div>\r\n	Madam A was a 48-year-old lady, widowed since 20 years ago and had two children out of consanguineous marriage. She was diagnosed with schizophrenia at the age of 28 years where she presented with second and third person auditory hallucinations and delusions of persecution and reference. Soon after the diagnosis, she developed marked depressive symptoms with active suicidal ideation secondary to the frightening psychotic experiences.</div>\r\n<div>\r\n	Since the first episode, she had never attained full remission. She had frequent relapses requiring hospitalizations partly due to her poor insight leading to non-adherence to medications and at times triggered by stressors present in the family. She was treated with a series of antipsychotics including haloperidol, chlorpromazine and, risperidone with suboptimal response. Her mother, Madam D, claimed to supervise her medications. However, it could not be ascertained as she was frequently distressed having to care for Madam A and two other sick family members. Madam A was once tried with clozapine with total dose of 700 mg daily which yielded better clinical response. However, due to her erratic intake of this oral medication, in addition to the risks of severe side effects the medication may pose, it was switched to intramuscular depot flupentixol and tablet olanzapine which resulted in similar satisfactory response. Unfortunately, olanzapine was later substituted with the cheaper haloperidol due to financial reason after her Parkinson-stricken father passed away with the termination of financial benefit which he used to receive from the Welfare Department.</div>\r\n<div>\r\n	Electro-convulsive treatment (ECT) was once commenced during one hospitalization and was continued on maintenance basis. However, she defaulted mainly due to the difficulty that the mother had to bring her to the hospital.</div>\r\n<div>\r\n	She was referred to the Community Mental Health Team (CMHT) of Universiti Kebangsaan Malaysia Medical Center (UKMMC) since last seven years. Through regular home visits of weekly to monthly by the team, her medications were monitored more closely and the family was helped to overcome the financial problem by engaging the relevant agencies. These services helped to gradually reduce her hospital admissions from four times a year to an average of once a year.</div>\r\n<div>\r\n	<div>\r\n		MISS B</div>\r\n	<div>\r\n		Miss B, the younger sister of Madam A; a 44-year-old single lady, first presented to UKMMC at the age of 22 years and was diagnosed with mental retardation with schizophrenia. She had history of multiple psychiatric admissions with typical presentation of florid auditory hallucinations and disorganized behaviour, causing significant distress to the family. She also received similar services from the CHMT which helped to stabilise her condition. Her last admission was four years ago. Her symptoms were controlled by sulpiride 200 mg bid and IM fluphenazine 25 mg, monthly.</div>\r\n	<div>\r\n		MISS C</div>\r\n	<div>\r\n		Miss C, the eldest daughter of Madam A, currently 24-years-old, was diagnosed with mental retardation since younger days. She was first hospitalized when she was 19 years due to psychosis and disorganized behavior which disrupted the family&rsquo;s function, as she needed extra care. She subsequently required frequent admissions two to three times a year when she relapsed with similar presentations. She was diagnosed with mental retardation and schizophrenia. Her symptoms were well controlled when she was on olanzapine. However, due to financial constraint; olanzapine was substituted with sulpiride at one stage, which had destabilized her illness with impending relapse. The CMHT managed to secure financial aid to sustain her previous medication. Currently she is doing well on olanzapine 15 mg nocte. With regular supervision of her medication by the CMHT, she had maintained to be symptom free and had not required hospitalization for a long time. She also regularly participated in group psychosocial rehabilitation activities scheduled by the team which provided the family some respite from caring for her.</div>\r\n	<div>\r\n		MADAM D</div>\r\n	<div>\r\n		She was a 68-year-old lady and a widow, who out of consanguineous marriage was blessed with two daughters; Madam A and Miss B. She was the only mentally healthy care-taker in the family; who had to care for her mentally ill daughters, grand-daughter and husband who suffered from Parkinson disease for many years before his death. She developed major depressive disorder seven years ago with family wellbeing and financial issues being her constant worries. She also suffered from long-standing diabetes mellitus, hypertension and had a right lacunar infarct four years back. She had a few admissions to the Medical Ward, due to the complications of her medical illness but had never been admitted to the Psychiatric Ward.</div>\r\n	<div>\r\n		Treatment with escitalopram 15mg nocte helped to control her symptoms. However, she would still have bouts of low mood when faced with difficulties in caring for her mentally ill family members. In view of her family&rsquo;s condition, she felt stigmatized and isolated from the community. She took care of the household and managed her mentally ill family members. The CHMT had worked with her in creating medication charts to improve medication adherence. The team was also vigilant in monitoring her mood and giving her emotional and practical support. This involved mobilizing support from relatives and relevant agencies.</div>\r\n	<div>\r\n		DISCUSSION</div>\r\n	<div>\r\n		This report demonstrated the complexity of managing a family with its households suffering from mental illness and what ACT could provide in restoring remission and improving their quality of life. ACT which is the earliest available component of community-oriented services in Malaysia, functions to integrate and organize psychiatric care for patients with severe mental illness to cater for their different needs at different stage of life (Ruzanna &amp; Marhani 2008). As demonstrated in this case, ACT has been proven in one study to reduce hospitalization, retain contact with service, improve patient satisfaction, and reduction of illness severity (Lambert et al. 2010). In accordance with the latter finding, a local study showed a high prevalence of symptomatic remission among schizophrenia patients who received ACT (n: 118, remission rate 78%) (Dahlan et al. 2013). In keeping view with all these facts, ACT should be provided to patients who are at high risk of repeated hospitalization and homelessness (Dixon et al. 2010).</div>\r\n	<div>\r\n		In the present case, ACT helped one entire family in their journey of living with mental illness. ACT could be an essential psychiatric service component that should be established at all localities.</div>\r\n</div>\r\n', 1),
(1264, 1256, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	First described by Dr. Nicholls almost 200 years ago, acute aortic dissection has become a feared cause of acute severe inter-scapular or chest pain radiating to the back (Hirst et al. 1958). Acute aortic dissection refers to a sudden split of the intimal layer occurring within two weeks of onset of symptoms, while the term chronic aortic dissection is reserved for dissection that persist for more than two weeks. The speed, lethality and rarity of this clinical entity warrants high index of suspicion in order to execute prompt and definitive treatment. Seen largely in men above the age of 60 years old (Hagan et al. 2000), aortic dissection can be divided into Stanford A and B depending on region of dissection. Stanford A dissection involves the ascending aorta presents more commonly with a higher mortality rate, while Stanford B involves dissection distal to the left subclavian artery (Golledge &amp; Eagle 2008). Unlike in the older age group where trauma, atherosclerosis and previous history of arterial dissection or aneurysm have been implicated as prominent risk factors, the rare occurrence of aortic dissection in the younger age group has been associated with trauma, Marfan&rsquo;s syndrome, uncontrolled essential hypertension, connective tissue disease, bicuspid aortic valve disease, cocaine abuse and during peri-partum period (Januzzi et al. 2004). Recent advances in medical technology have paved the way for less invasive methods of treatment such as endovascular aortic repair. The choice between endovascular and open repair would involve a broad spectrum of criteria extending from clinical to radiological. We aim to highlight an interesting case of multiple synchronous acute arterial dissections occurring in a 32-year-old male.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 32-year-old male presented to the Emergency Department with sudden onset of excruciating pain of the inter-scapular region for the past 12 hours. He described the nature of pain as tearing and stabbing. The initial onset of pain occurred while he was resting in bed. The pain was not relieved by oral analgesics and had increased in severity over the past two hours. He denied prior medical illness, recent trauma or cocaine abuse. At the triage, he was noted to be hypertensive with blood pressure of 217/116mm Hg. His resting heart rate was around 90 beats per minute with oxygen saturation of 98% under room air. Clinical examination of his cardiovascular, pulmonary and gastrointestinal systems was otherwise unremarkable. Echocardiogram revealed no evidence of cardiac ischemia, however chest radiograph showed significant mediastinal widening. On further questioning, he denied experiencing weakness of limbs, claudication or rest pain. Detailed examination of the peripheral pulses revealed an audible bruit over the right femoral artery with monophasic Doppler signals heard over the right femoral artery and arteries distal to it. Capillary refill time, motor and sensory examination of all limbs was satisfactory. Intravenous infusion of antihypertensive agents was commenced with close cardiac monitoring. An urgent computed tomographic angiography (CTA) of the thorax, abdomen and lower limbs was requested (Figure 1a). CTA revealed multiple areas of arterial dissection. A 9.4 cm Stanford B dissection and another separate arterial dissection extending from the bifurcation of the right common iliac artery to the right superficial femoral artery were noted. An isolated small fusiform aneurysm was noted close to the origin of the left subclavian artery measuring approximately 1 cm in diameter.</div>\r\n<div>\r\n	A staged procedure involving open surgery followed by endovascular repair of the aortic dissection was performed. The first stage involved sternotomy and placement of interposition bypass graft between the left common carotid and second part of the left subclavian artery. This was followed by plugging of the origin of the left subclavian artery by interventional radiology. During surgery, the wall of the left subclavian artery was noted to be friable. Biopsy specimen of the left subclavian artery wall was taken to rule out connective tissue disease. Two days later, thoracic endovascular aortic repair of the Stanford B dissection was carried out as part of stage 2 management of this patient. Chest radiograph done following TEVAR confimed position of the endovascular stent (Figure 1b). Immidiate post-operative recovery was unremarkable. Patient however seccumbed on day 5 of TEVAR following cardiopulmonary arrest secondary to acute myocardial infarction. Biopsy result from left subclavian arterial wall was negative for connective tissue disease.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Hallmark of aortic dissection is intimal injury. Intimal injury permits pulsatile high-pressured blood flow to track between the tunica intima and tunica media. The passage of blood between these two layers raises an intima-medial flap by splitting the tunica media into an inner layer that is also known as the intima-medial layer and an outer layer. In some cases, this process is augmented by pre-existing medial cystic degeneration. High-pressured passage of blood beneath between the two layers of tunica media gives rise to a false lumen, which is nothing more than a blind track. A large intimal tear leads to a larger false lumen. Secondary tears in the tunica intima can occur due to re-entry of blood into the true lumen from the false lumen (Roberts &amp; Roberts 1991).</div>\r\n<div>\r\n	Exact pathophysiology of intimal tear has been a topic of much debate. Rupture of atherosclerotic plaque or plaque ulceration, intimal injury due to uncontrolled hypertension, connective tissue disorder and direct iatrogenic injury have all been implicated as causative factor for that matter. However, aortic dissection secondary to intramural hematoma (IMH) has a slightly different IMH occurs secondary to rupture of vasa vasorum. The blood within the media, splits the tunica media layer and give rise to a false lumen. Although there is no direct communication between the true lumen and false lumen in the case of aortic dissection secondary to intramural hematoma, the mainstay of treatment remains the same (Roberts &amp; Roberts 1991; Svensson et al. 2002).</div>\r\n<div>\r\n	The rarity, varying as well as non-specific clinical presentation of aortic dissection often leads to a missed diagnosis (Khan &amp; Nair 2002). Left untreated, one in for four will die within the first 24 hours and mortality rates could reach up to 90% within two weeks from onset of symptoms (Sommer et al. 1996). The most common presenting symptom is excruciating back pain. Other symptoms include abdominal pain, syncope and weakness or numbness of extremities. The pain is most severe at the beginning and is often localized to the site of intimal tear. Intimal tear that is distal to the left subclavian will manifest as chest pain radiating to the back where else a tear above the aortic valve will present as anterior chest pain. Dissection of thoracoabdominal aorta often presents as lower back or left flank pain. Spread of pain is indicative of progressive dissection (Ledbetter et al. 1999; von Kodolitsch et al. 2000). Painless dissection has been documented to occur in a small percentage of patients. Such asymptomatic dissections tend to involve the elderly, individuals on long-term steroids and those diagnosed with Marfan&rsquo;s Syndrome (Svensson &amp; Crawford 1992). Focal neurological deficits are seen with involvement of the great vessels Hemiparesis occurs following poor perfusion to the spinal vasculature. Sycope is seen following hemorrhage into the pericardial space in proximal dissection. Hemopericardium causes pericardial tamponade that may manifest as syncope or even sudden death. Dissection of the thoracoabdominal aorta is often implicated in causing malperfusion of the kidneys, mesentery and lower limbs. In such instance, chief complaint would be severe abdominal pain and symptoms suggestive of lower limb ischemia such as claudication and rest pain. Perfusion deficit in these organs can result as a consequence of obstruction due to the dissection flap either by dynamic obstruction or static obstruction. Dynamic obstruction is due to prolapse of dissection flap across a visceral artery. Static obstruction occurs when there is dissection extending into the visceral vasculature. Post obstructive arterial thrombosis, embolism, aortic valve distortion leading to severe cardiac failure are other causes that have been identified as cause for end organ damage (Williams et al. 1997). Hypertension is seen as a common feature of distal dissection, occurring in almost 70% of cases. On the contrary, only 36% of proximal dissection presents with hypertension. Approximately 20% of Standford A dissection is associated with hypotension secondary to severe aortic insufficiency, cardiac tamponade and myocardial infarction (Klompas 2002). Pseudohypotension is another entity that one should be aware of. Pseudohypotension occurs when there is compromise to branchial artery circulation following proximal dissection (Sheikh et al. 2013).</div>\r\n<div>\r\n	Echocardiograph is often the initlal investigative tool. Presence of an elevated ST wave or inverted T wave denotes acute myocardial ischemia (Klompas 2002). Widened mediastinum on chest radiograph suggests aortic dissection. Such a finding however warrants a more definitive imaging modality with higher sensitivity and specificity. CTA has sensitivity of 99% and specificity of almost 98% in detecting aortic dissection. Magnetic resonance Imaging (MRI) is more specific than CTA with ability to differentiate between penetrating atherosclerotic ulcers from intramural hematoma. However, the long duration required for image acquisition renders it less favourable (Zeman et al. 1995; Shiga et al. 2006). Negative finding on radiological imaging in the presence of high clinical suspicion of an acute aortic dissection warrants application of a second imaging modality (Svensson et al. 1999). Raised serum urea, creatinine and lactate suggest ongoing visceral hypoperfusion and end organ damage.</div>\r\n<div>\r\n	Treatment of acute aortic dissection would depend on site of dissection, severity of presentation and concomitant existence of life threatening complications (Hiratzka et al. 2010). Stanford A acute aortic dissections requires urgent surgical evaluation for emergency surgical repair in view of it&rsquo;s high risk of rupture. Open surgery is preferred over endovascular repair in the setting of acute proximal aortic dissection, requiring excision of proximal extent of the dissection and replacement with Dacron graft (Hagan et al. 2000). Stanford B acute aortic dissection on the other hand can be managed conservatively through medical management. Due to a lower pressure of blood flow through the false lumen in distal dissection as compared to proximal aortic dissection, further progression of the dissection often comes to a halt due to thrombosis of the false lumen. Use of beta-blockers and angiotensin converting enzymes has been proven to control hypertension and limit progression of distal aortic dissection. However, progression of dissection, malperfusion syndrome, enlargement of co-existing aortic aneurysm warrants prompt surgical intervention (Estera et al. 2006). Transthoracic endovascular aortic repair (TEVAR) is the preferred surgical treatment option in complicated Stanford B aortic dissection. Studies show a reduction in mortality and paraplegia rates with TEVAR as compared to conventional open surgery (Leurs et al. 2004; Eggebrecht et al. 2006; Chen et al. 2006).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Our patient was diagnosed with acute Stanford B aortic dissection. Decision for surgical intervention was carried out due to the close proximity of the dissection with the origin of the left subclavian artery. Left untreated, it could have led to impaired perfusion of the entire left upper limb. Decision for TEVAR was in accordance to recommended guidelines, however the risk of iatrogenic occlusion of the left subclavian artery outweighed the benefit in that setting and prompted us to proceed with an interposition bypass graft between the left common carotid and left subclavian prior to embarking on endovascular repair. The non-complicated distal aortic dissection in our patient was managed conservatively. The cause of dissection in our patient remains unknown. Acute aortic dissection is a life threatening condition and failure to identify and administer appropriate treatment can be fatal. Thus, it is imperative that surgeons know of this rare clinical entity. The non&ndash;specific and broad spectrum of clinical presentation poses a diagnostic challenge. A combination of atypical chest pain, ischemic pattern on ECG, mediastinal widening on chest radiograph and deranged renal profile as well as elevated serum lactate should increase the likelihood of aortic dissection. Decision to intervene surgically or continue with medical management should depend on CTA finding, progression of dissection and evidence of life threatening complications.</div>\r\n', 1),
(1267, 1259, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Orogastric lavage was previously a common method for gastric emptying in the emergency department (ED). The frequency of its use has been decreasing over the past decade (Larkin &amp; Claassen 2007). This is due to the complications that arise and the clinical efficacy of this method. However, given the correct selection of patient and the proper technique and safety profile, this method can be lifesaving</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	An 18-year-old girl was brought into the Emergency Department with vomiting and reduced consciousness by her family members. According to her mother, she had consumed a clear liquid form insecticide, which was kept in a clear plastic bottle at home. It is known that she was upset with the recent death of her boyfriend. She ingested the toxin 20 minutes prior to arrival.</div>\r\n<div>\r\n	Upon arrival, she was brought into the red zone. She had an unspecified unpleasant odour. She was breathing spontaneously but in a labored manner. On examination, her vital signs included a temperature of 36.8&deg;C, pulse rate of 100 beats/min, blood pressure 121/62 mmHg, respiratory rate 30 breaths/min and oxygen saturation of 96% under room air. She had flexion to painful stimuli and did not open her eyes nor make sounds. Her Glasgow Coma Scale (GCS) score was 6/15. She had pinpoint pupils. Lung auscultation revealed bilateral transmitted sounds. Examination of the heart and abdomen was unremarkable. She was passing large amount of loose stool. The skin was warm and clammy with capillary refill (CR) of less than two seconds. She soiled the bed linen.</div>\r\n<div>\r\n	After orotracheal intubation and mechanical ventilation was performed for airway protection, intravenous atropine was administered (0.02 mg/kg every five minutes) and pralidoxime was initiated. At this point, intoxication with an organophosphorus compound (OP) was considered due to the toxidromes present, which were diarrhoea, pin point pupils, bronchorrhea and confusion. In view of early presentation, in the time frame of one hour from the time of alleged consumption, orogastric lavage was done. She was also given activated charcoal via orogastric tube</div>\r\n<div>\r\n	During orogastric lavage the patient is sedated and placed at lateral decubitus position. The patient was already intubated, hence pulmonary aspiration will no longer be a problem. The placement was confirmed with air insufflations and auscultations. Instantaneously approximately 50 cc of green-yellowish fluid with toxic (organophosphate) fluid was drained from the tube. Five hundred mililiters of water was added in the tube and siphoned out again draining remnants of the organophosphate toxin.</div>\r\n<div>\r\n	The patient was admitted to the Intensive Care Unit (ICU) and was extubated the following day. All laboratory values were within normal range except a high white cell count of 16.4 x 109/L. The white cell count normalized to 5.5 x 109/L four days later. The cholinesterase level was not available. She was referred to the internal medicine team. She was discharged well with no complications five days later.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Orogastric lavage is used to remove pills, fragments and toxic material from the stomach. The frequency of orogastric lavage has been reducing in the past decade. There is a downward trend over the last decade in the usage of gastric lavage in the US (Benson et al. 2013). It is seldom done to the fear of risk of complications such as insertion of tube in the trachea, aspiration, laryngospasm, arrhythmia, esophageal or stomach perforation, fluid/ electrolyte imbalance and decreased oxygenation during procedure (Thompson et al. 1987). Many organizations, including the American Board of Applied Toxicology and the Canadian Association of Poison Control Centre has warned against the use of orogastric lavage.</div>\r\n<div>\r\n	The claim that orogastric lavage is unsafe should be reviewed. Certain guidelines amongst airway protection is mandatory should be drafted on this procedure. The cited consensus article updates the data on the basis of animal studies, studies of volunteers, experimental studies in patients, case studies, and clinical studies. Animal studies and experimental studies fail to represent the real clinical scenario. Nevertheless, several case reports showed positive outcomes for gastric lavage and endoscopy. With regard to clinical studies the cited studies are from 1942 to 1966 and did not consider the intoxication dose and the factor of time. Further citations (review of 56 small and heterogeneous, exclusively Chinese, studies) produce a controversial picture. Although benefit of lavage is immediate return of pill fragments and prevention of further intoxication, studies demonstrate drug removal ranging from 35 to 56 percent. Two out of three large studies showed benefit from the procedure, only if performed within one hour of ingestion. (Merigian et al. 1990; Pond et al. 1995)</div>\r\n<div>\r\n	In cases of life-threatening intoxication, all possible options for detoxification should be considered. Gastric lavage is one such option. In such a situation, a benefit not supported by controlled studies should not be a deterrent factor against gastric lavage. Time factor should still be of a consideration, as of theoretically toxins would be cleared from the stomach within one hour. This also depends on the type of toxins, such as toxins that delays gastrointestinal motility and clearance (e.g. tricyclics) or toxins that have enterohepatic circulation (salicylates, paracetamol) should have a longer time frame in which lavage can be done. The physical nature of toxins should not be questioned, as in this case liquid form of the organophosphate was able to be siphoned out. We have repeatedly encountered situations where the lack of, or delay in, gastric lavage resulted in poor outcomes.</div>\r\n<div>\r\n	Deliberate self-poisoning is significantly more dangerous in the developing world than in the West. Orogastric lavage would have poor outcomes if done as reported in underdeveloped countries with no ICU setting or antidote availability. It was reported that gastric lavage was done forcefully with no sedation, no airway protection and no proper monitoring. In addition to this, toxic substances that are more dangerous are widely available in developing countries (ie paraquat). The fatality of less than 0.5% in western world contrasts strongly with 10-20% in the developing world. (Eddleston et al. 2007). Lack of experience (Benson et al. 2013) is another contributory factor in the reducing trend of orogastric lavage performed. Since the range selection of patients that lavage can be performed is small, it is rarely done and few, if any new freshly graduates have neither any experience in perfoming or witnessing an orogastric lavage being done.</div>\r\n<div>\r\n	In Malaysia, where ICU setting is modernized, and facility to intubate and protect the airway in the Emergency Department is available, in addition with the widespread use of cardiostable drugs (ie etomidate) it is safe to intubate and protect the airway of a patient with fatal toxic ingestion prior to orogastric lavage. In view of this scenario, we have outlined a few recommendations based on previous literature in performing an orogastric lavage</div>\r\n<div>\r\n	RECOMMENDATIONS</div>\r\n<div>\r\n	Selection of patients must be of time frame within one hour on ingestion. Amount and toxicity of substance ingested is perceived fatal, based on history and toxidromes obtained at that moment. Airway protection is mandatory, as patients will be intubated following the rapid sequence intubation (RSI) protocol. Staff and medical personnel should be protected and all should have the necessary personnel protective equipment (PPE).</div>\r\n<div>\r\n	The orogastric tube must be of a correct diameter (36-40 french tube in adults and 22-24 &ndash; French tube). The tube must be correctly measured from the chin to the xiphoid process. Insufflation of air and auscultation would ensure accurate placement of the tube. Siphon out any toxic substance that comes out as soon as the tube is inserted. Lavage with room temperature water until effluent becomes clear. Before removing the tube, instil activated charcoal in dose of 1g/kg (Vale &amp; Kulig 2004).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Orogastric lavage has fallen out of favour for the past decade due to complications, poor patient selection, poor technique and lack of experience. The benefits of orogastric lavage are yet to be elucidated through proper randomized controlled trials. This procedure should be limited to patients with presumed fatal intoxication and within the time frame of gastric emptying of 1 hour. Proper technique, adequate monitoring and securing definitive airway protection are necessary for safe orogastric lavage. Direct benefit in reducing toxin ingestion up to 50% could be lifesaving in conditions where the risk of this procedure is minimized.</div>\r\n', 1),
(1269, 1261, NULL, NULL),
(1275, 1267, '<div>\r\n	QUESTION</div>\r\n<div>\r\n	A 4-year-old child was brought to the Emergency Department with generalized</div>\r\n<div>\r\n	tonic-clonic seizures. Right hemiparesis and unequal pupils were noted during examination. Computed Tomography of brain revealed acute subdural hemorrhage for which an emergency craniotomy and evacuation of clot was performed. Intraoperative finding of abnormal vessels prompted a need to revisit his past surgical history. Three years ago, he was diagnosed with hydrocephalus secondary to cerebral aqueduct stenosis, which eventually led to the insertion of a ventriculo-peritoneal shunt. Since then, he has suffered from global developmental delay and infrequent episodes of epilepsy. One month prior to this recent presentation, he was treated for varicella zoster infection and discharged well, five days later. The initial Magnetic Resonance Imaging of brain taken three years earlier is also shown in Figure 1.</div>\r\n<div>\r\n	What should be the more appropriate cause of hydrocephalus in this patient?</div>\r\n<div>\r\n	Is there a correlation between varicella zoster infection and the current presentation of acute subdural hemorrhage?</div>\r\n<div>\r\n	Mail your response to the editorial address (medhealthukm@gmail.com). Answers will be published in the 2015(1) issue of Medicine and Health Journal.</div>\r\n', 1),
(1279, 1271, NULL, NULL),
(1290, 1282, NULL, NULL),
(1293, 1285, NULL, NULL),
(1299, 1291, NULL, NULL),
(1301, 1293, NULL, NULL),
(1308, 1300, NULL, NULL),
(1309, 1293, NULL, NULL),
(1316, 1307, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Acne is a common skin disorder which has great impact on individuals as it affects physical appearance, psychological function and impairs quality of life. (Katsambas &amp; Lotti 2003; Koo 1995). Even the mild grade of acne may affect function and diminish quality of life (Lasek &amp; Chren 1998; Picardi et al. 2000). Although specific treatment for acne is available, self-management is often the first step taken to overcome acne problems. Products such as facial cleansers, peeling agents, creams, and oral contraceptive for female are some of the favourites. (Lasek &amp; Chren 1998; Goncalves et al. 2012). Other measures include increased water intake, using non-prescriptive topical agents and squeezing acne lesions (Ali et al. 2010).</div>\r\n<div>\r\n	It is a common assumption that medical students would be more inclined to get themselves treated as they have the knowledge about medical conditions and the access to treatment facilities. Yet, how many of these future doctors actually seek treatment and how acne impacts their life remains a mystery. The aim of the present study was to assess disability, self management and help-seeking behaviour for acne among medical students.</div>\r\n<div>\r\n	Materials and Methods</div>\r\n<div>\r\n	This study was approved by the Universiti Kebangsaaan Malaysia Ethics Committee.&nbsp;</div>\r\n<div>\r\n	A cross sectional study was conducted over a period of 12 months, at the Universiti Kebangsaan Malaysia (UKM), Kuala Lumpur. Medical students from academic years 1 to 5 were selected using stratified cluster random sampling. A total of 400 medical students were invited to participate and 361 consented. Participants were recruited through short message services (SMS), phone calls and social network. &nbsp;Students were assessed for acne severity using Comprehensive Acne Severity Score (CASS). &nbsp;This is a validated acne grading system to assess acne severity on the face, chest and back using scores ranging from 0 to 5: Score 0 (Clear), score 1 (almost clear), score 2 (mild), score 3 (moderate), score 4 (severe) and score 5 (very severe). &nbsp;Score of 0 is considered as absence of acne while scores 1 to 5 as presence of acne (Tan et al. 2007).<span class="Apple-tab-span" style="white-space:pre"> </span></div>\r\n<div>\r\n	Students diagnosed with acne were given the Cardiff Acne Disability Index (CADI) and Acne Management Questionnaire. &nbsp;CADI is a 5 item questionnaire, used for measuring the quality of life among teenagers and young adults with acne (Motley &amp; Finlay 1992). These questions assess the impact of acne over the last month:&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	a)<span class="Apple-tab-span" style="white-space:pre"> </span>Emotional disability:</div>\r\n<div>\r\n	Question <span class="Apple-tab-span" style="white-space:pre"> </span>1 (As a result of having acne, during the last month have you been aggressive, frustrated or embarrassed?)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	b) Social disability:&nbsp;</div>\r\n<div>\r\n	Question 2: (Do you think that having acne during the last month interfered with your daily social life, social events or relationship with members of the opposite sex?) and question 3 (During the last month have you avoided public changing facilities or wearing swimming costumes because of your acne?)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	c) Feelings on appearance: &nbsp;</div>\r\n<div>\r\n	Question 4 (How would you describe your feelings about the appearance of your skin over the last month?)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	d) Perceived severity:&nbsp;</div>\r\n<div>\r\n	Question 5 (Please indicate how bad you think your acne is now)&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	For each question, there were four response categories assigned to scores ranging from 0 to 3 scores with a minimum total score of 0 and a maximum of 15. &nbsp;A total score of 0 to 5 was classified as mild disability, scores 6 to 10 as moderate disability, while 11 to 15 as severe disability.<span class="Apple-tab-span" style="white-space:pre"> </span></div>\r\n<div>\r\n	The Acne Management Questionnaire was used to assess student&rsquo;s self-management and help seeking behavior. This is a self-administered questionnaire containing a total of six questions. Each question had few answer options and a blank space for any additional information (Smithard et al. 2001).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	a) Self-perception on acne</div>\r\n<div>\r\n	(Question 1: Do you think you have acne at the moment?)&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	b) Acne self-treatment (Question 2: Have you ever done any of the following to improve the appearance of your skin? (List of options provided) &amp; Question 3: Do you apply any lotion or cream to help prevent spots?)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	c) Help-seeking behavior (Question 4: Who do you talk to about any problems you may have with the spots? &amp; Question 6: Do you seek help if you had acne?)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	d) Knowledge on acne (Question 5: Do you think acne is a condition that can be cured?).</div>\r\n<div>\r\n	Data analysis&nbsp;</div>\r\n<div>\r\n	Statistical Package for Social Sciences (SPSS, version 20.0) was used for data analysis. Descriptive statistics were used to analyze frequency, percentage, means, and standard deviation of the appropriate variables. Student T test was used to determine association between gender, ethnicity and CADI; Pearson Chi-square was used to determine association between help-seeking behaviour, gender and ethnicity. Significance set at p&gt;0.05 and confidence level (Cl) of 95%, spearman correlation was used to determine association between CADI and CASS.&nbsp;</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	A total of 361 students completed clinical examination, CADI and acne management questionnaires. Acne prevalence was 68.1% (n=246). &nbsp;The assessment of acne severity using the CASS revealed that majority (55.7%, n=137) had almost clear skin followed by mild (35.0%, n=86), and moderate (7.7%, n=19) grades of acne. Only a small percentage (1.6%, n=4) had severe acne while none of them had very severe acne. Male to female to female ratio was 1:1.1.</div>\r\n<div>\r\n	The mean CADI disability score was 3.35 + 2.39. The levels of impairment were mostly mild (83.8%, n=206) followed by moderate (14.6%, n=36) and severe (1.6%, n=4). Feeling about appearance domain had the highest mean scores (1.05 + 0.77) (Table 1). Females had significantly higher disability scores compared to males (3.76 + 2.24, p=0.006) especially in the domain of appearance (1.20 + 0.72, p=0.001) and perceived severity (1.09 + 0.64, p&lt;0.05). Students from the Non-Malay ethnic group also had significantly higher mean disability compared to the Malay ethnic group (3.79 + 2.59, p=0.018) (Table 2). There was no correlation between CADI and CASS for all grades of acne severity.&nbsp;</div>\r\n<div>\r\n	Among students with acne, majority (84.6%, n=208) correctly identified themselves to have this condition however 15.4% (n=38) of students perceived that they did not have acne. Regarding the self management of their acne, students washed their face more often (73.2%, n=180) while 72.4% (n=178) drank more water. Most students 87.4% (n=215) knew that acne can be cured. However, about 12.6% (n=31) were unsure or did not know that acne can be treated. Almost half of the students (54.1%, n=133) with acne preferred talk to friends about this problem, while only 9.3% (n=23) preferred to consult physicians. Although medical students had acne, they were not keen to seek help for their condition as they did not perceive it as a problem (43.9%, n=108) (Table 3). Female students preferred to seek help from siblings (70.3%, p&lt;0.05) while males preferred consults doctors (69.6%, p = 0.033) (Table 4).</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Acne impairs quality of life and this has been well established in the literature (Law et al. 2009; Kokandi 2010). The impact of acne on medical students in addition to their academic and professional stress may further impair the emotional and mental stability of these future doctors.&nbsp;</div>\r\n<div>\r\n	The disability caused by acne among medical students in this study is categorized as mild with the mean CADI score of 3.35 + 2.39. This was similar to a study among university students in Hong Kong (mean CADI score of 2.56, Lasek &amp; Chren 1998). This is possibly due to the fact that most of these students have less severe acne and did not actually seek treatment for acne. The disability of acne is expected to be higher among those who seek treatment for acne. This was shown in a recent study by Yap where acne disability was found to be higher 5.1 + 3.83, (Yap 2012). The current study also did not demonstrate any correlation between CADI and acne severity. It is postulated that patients seeking treatment for acne would have more disability compared to those who don&rsquo;t. A study in Hong Kong showed significant association between acne severity and impairment for those with mild but not for those with moderate or severe acne.</div>\r\n<div>\r\n	Female students in this study report greater disability due acne especially regarding their perceived severity and feeling about their appearance compared to males. &nbsp;Earlier studies depict a controversy between disabilities and gender (Law et al. 2009; Yap 2012). Different age groups probably relate to acne differently with young adults being more concern with appearances as they are at the stage of life where they form new relationships. This study also showed that the non-Malays report greater disability compared to the Malay ethnic group which suggests that different cultural believes and tolerance for disorders affects the overall perception of their appearance. &nbsp;</div>\r\n<div>\r\n	Most of the medical students in this study correctly identified the fact that they had acne however a small percentage (15.4%) denied having acne implying that they may either be in denial or may not recognize acne. Same result was reported among Australian school students but the earlier study subjects were school students. Hence, they may lack the knowledge regarding acne (Kilkenny et al. 1998).</div>\r\n<div>\r\n	Surprisingly about 12.6% of medicals students were not aware that acne can be treated. This is contrary to the general expectation that medical students would be more concern about medical disorders relevant to them. In fact, only a small percentage of them stated that they would consult a physician for their acne while others would seek non-formal sources of treatment. &nbsp;They tend to self manage their acne by washing their face more often (73.2%) and drinking more water (72.4%) although none of these measures have been shown to be effective based on the available evidence. A few other studies have also shown that that drinking water was believed to improve the appearance of skin (Smithard et al. 2001; Suh et al. 2008).</div>\r\n<div>\r\n	When seeking for help, most students choose to talk about their acne problem with their friends (54.1%), parent (34.1%) and siblings (30.1%) rather than consulting a doctor, despite the waiver on consultation and treatment fee. Students preferred to share their problems based on familiarity rather than seeking actual professional help such as doctors although they are in the same environment. This could be due to the lack concern for acne as almost half of them did not consider acne as a problem (43.9%). Other studies also reported similar findings (Cheng et al. 2010; Tan et al. 2007).<span class="Apple-tab-span" style="white-space:pre"> </span></div>\r\n<div>\r\n	The overall help-seeing behaviour of medical students for their acne is acceptable. However, it requires modification as acne is a treatable condition. Delay or deferring adequate treatment may result in unnecessary complications such as scarring (Newton et al. 1997). Hence, awareness regarding acne and its management needs to be promoted to prevent permanent scaring and disfigurement.</div>\r\n<div>\r\n	The limitation of this study includes the possible discrepancy in grading acne severity among the researchers, although they received hands-on training on diagnosing acne and using the CASS. Another limitation is that participants with severe acne in the past may have consulted a physician however this information would not be captured as the participants were assessed on their current status.</div>\r\n<div>\r\n	Conclusion&nbsp;</div>\r\n<div>\r\n	This study shows that a high percentage of young adults are affected by acne. However, this study failed to demonstrate any relationship between acne severity and quality of life. &nbsp;Although the overall impact of acne on quality of life is mild, it has greater implication on females and the Non-Malay ethnic group suggesting that this group may require screening and proactive management for acne. &nbsp;The lack of knowledge regarding acne and its management among the medical students may be addressed during their medical training in dermatological disorders. Strengthening their knowledge on evidence based management may be able provide them the confidence that acne can be treated effectively. Their help-seeking behavior for acne requires modification as the opportunity for consultation and treatment are easily accessible for medical students. Adequate treatment can prevent permanent scars which may affect quality of life of these future doctors. The reluctance for a formal consultation and treatment among medical students requires further evaluation and a qualitative design study could be more informative.&nbsp;</div>\r\n<div>\r\n	Acknowledgment</div>\r\n<div>\r\n	The authors would like to thank Universiti Kebangsaan Malaysia for funding this project. The authors also thank Prof. Dr. Cris Glazebrook, from Queens Medical Centre, Nottingham for the permission received to use the acne management questionnaire and Prof. Dr. Jerry K.L. Tan from University of Western Ontario, Canada, for the permission received to use the Comprehensive Acne Severity Score (CASS).</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1322, 1313, '<div>\r\n	Core needle biopsy (CNB) is widely used in routine preoperative practice as part of the triple assessment in patients with suspected breast carcinoma (Li et al. 2012; Wai et al. 2013). Studies have reported good concordance rate of 91%-100% between CNB and excisional biopsy (EB) for diagnosis of breast carcinoma with a specificity rate ranging from 96% to 100% (Li et al. 2012).&nbsp;</div>\r\n<div>\r\n	&nbsp;Immunohistochemical hormonal receptor (HR) markers such as estrogen receptor (ER), progesterone receptor (PR) and HER2 performed on tumor samples from CNB has been used as biologic markers in breast cancers for predicting response to specific therapeutic agents. Similarly, HER2 over expression has been associated with worse prognosis in newly diagnosed patients with breast carcinoma, and is a determinant of response to trastuzumab (Arnedos et al. 2009). However, challenges may arise as analysing HR markers on CNB containing small tissue samples may not accurately reflect the overall biologic profile of the heterogenous tumour. Thus, HR markers on CNB may lead to false negative or positive results. When &ldquo;false negative&rdquo; HR test results are found, these deprive women of the proven benefits of endocrine therapies and may lead to choosing unnecessary chemotherapy treatments with significant financial costs and toxicities (Uy et al. 2010).</div>\r\n<div>\r\n	In the light of false negative or false positive HR markers that may be found on CNB, the main aim of the present study was to compare the status of ER, PR and HER2 markers performed on CNB with the immunohistochemical results in the subsequent mastectomy/wide local excision/lumpectomy (EB) for patients diagnosed with breast cancer. More specifically, this study aimed to determine the level of concordance between hormonal status identified on CNB and EB performed in one of the local teaching hospitals in Malaysia.</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	Patients and samples</div>\r\n<div>\r\n	A retrospective cohort reviewing data and medical records of patients diagnosed with breast carcinoma from January 2002 until December 2012 in UKMMC was performed. The study was approved by the UKM Medical Centre Ethics Committee/Institutional Review Board (UKM Ethics Committee Reference No: UKM 1.5.3.5/244/FF-077-2013). A total of 107 female patients with CNB and subsequent EB diagnosed with primary breast carcinoma were included in this descriptive study. &nbsp;Patients who had local recurrent or non-primary carcinoma were totally excluded from this study. Patients who had received neoadjuvant radio or chemotherapy or hormonal treatment between CNB and the final excised specimens were also excluded from this study.</div>\r\n<div>\r\n	ER, PR and HER2 determination</div>\r\n<div>\r\n	Immunohistochemistry was used to determine ER, PR and HER2 hormonal status. ER and PR were graded using Allred score from 0 to 8 and scores of more than 2 were considered positive. HER2 was scored from 0 to 3+. Scoring of 2+ and 3+ in HER2 status analysis were taken as positive while 0 and 1+ was considered negative. The final results between CNB and EB were compared to determine the concordance and discordance rate.</div>\r\n<div>\r\n	Statistical analysis</div>\r\n<div>\r\n	The concordance rate, sensitivity, specificity, positive and negative predictive value were compared using SPSS version 19 by considering CNB as test assessment and EB as the gold standard.&nbsp;</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Concordance rate</div>\r\n<div>\r\n	A total of 93 samples were included in this study. A total of 76 patients from the 93 samples analysed, had infiltrating ductal carcinoma, diagnosed both on CNB and EB (81% respectively).&nbsp;</div>\r\n<div>\r\n	In CNB, PR was scored as positive in 65 (70%) and negative in 28 (30%) of the cases. In the EB, PR was positive in 62 (67%) of the cases and negative in 31 (33%). Discordance of PR status between CNB and EB was seen in 11 cases (11.8%).&nbsp;</div>\r\n<div>\r\n	ER status was 53% positive on CNB compared to 48% in the excised specimen. Only 12 samples were discordant (14%). 8 of the 12 discordant samples showed ER positivity on CNB but negative on EB. &nbsp;The remaining four discordant samples were ER negative on CNB but positive on EB.&nbsp;</div>\r\n<div>\r\n	Only 87 samples were available for HER2 status assessment. Fourty nine (56.3%) samples showed HER2 positivity on CNB and 38 (43.7%) samples were negative for HER2. &nbsp;In the EB, 39 samples were HER2 positive (44.8%) and 48 were negative (55.2%). A total of 26 samples were discordant with a discordant rate of 28.7%. Eighteen of these discordant samples showed HER2 positivity on CNB but were negative on EB. &nbsp;Summarized results of these findings were shown in Table 1.</div>\r\n<div>\r\n	Sensitivity and specificity of CNB and EB in detecting ER, PR and HER2 status in breast carcinoma</div>\r\n<div>\r\n	CNB was as sensitive as EB in determining ER and PR status. However, the sensitivity in determining HER2 on CNB was less consistent. &nbsp;The rates of sensitivity, specificity, positive and negative predictive values were summarized in Table 2 with EB as the reference for each marker.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	In routine preoperative practice in UKMMC, CNB is often used as part of a triple assessment in patients with suspected breast carcinoma. The expression of these markers will guide the Clinician in the therapeutic management of the patient. Previous studies concluded that immunohistochemical results from CNB tend to be less reliable than those analysed on excised specimens as CNB represent limited sample of tissue with possible tumour heterogeneity (Burge et al. 2006; Arnould et al. 2012).&nbsp;</div>\r\n<div>\r\n	CNB and EB samples from patients assessed in this study was not subjected to any mode of preoperative treatment, as treatment have been known to alter the tumour biologic marker such as ER, PR and HER2 (Honkoop et al. 1997). However, the tumour marker alterations seen in treated specimens may not be consistent with HER2. More recently, HER2 status was shown to remain unchanged when preneoadjuvant and post neoadjuvant specimens are compared (D&rsquo;Alfonso et al. 2010).</div>\r\n<div>\r\n	Previous studies found concordance rates for CNB and surgically excised specimens range between 81.3% to 100% for ER, between 42% to 89% for PR and from 86.9% to 100% for HER2 (Burge et al. 2006; Sutela et al. 2008; Wood et al. 2007; Tamaki et al. 2010). In agreement with previous findings (Burge et al. 2006; Wood et al. 2007; Sutela et al. 2008; Ricci et al. 2012), the present study found good concordance between CNB and EB for ER and PR. Previous reports have also found that the concordance rate between CNB and EB for ER was higher than PR (Arnedos et al. 2009). However, in the present study, concordance rate between CNB and EB for PR was slightly higher than that seen for ER, possibly due to better fixation in the CNB specimen.</div>\r\n<div>\r\n	In the present study, discordance between CNB and EB for ER was 14% and this finding is consistent with a previous study (Connor et al. 2002). False negativity for ER was seen in four cases, indicating that all ER negative results on CNB should be further confirmed with EB. As previously highlighted by Connor et al. (2002), false positivity for ER observed in eight cases of this study suggest sampling error within the tumour periphery and possibly poor fixation in the excised specimen.</div>\r\n<div>\r\n	While comparing previous studies (Wood et al. 2007; Tamaki et al. 2010; Ricci et al. 2012), the concordance rate between CNB and EB for HER2 in this study was lower than that seen for ER and PR and with a much lower sensitivity and specificity rates. The less consistent concordance rate for HER2 was possibly due to limited number of samples analysed in this study as well as intratumour heterogeneity. Given the limitation of tumour diversity, the concordance rates appear to be dependent on the number of cores analysed in that the higher number of cores are analysed, the more concordant are the tumour markers between EB and CNB (Tamaki et al. 2010).</div>\r\n<div>\r\n	Although, good concordance rates between CNB and ER were found in the present study, all of these findings were not found to be significant, possibly due to several limitations that may have potentially resulted such as limited number of sampling. In addition, differences in immunohistochemistry assays and fixative procedure may have also influenced the findings of this study. Breast cancers with HER2 2+ on immunohistochemistry are considered equivocal and therefore should undergo fluorescence in situ hybridization (FISH) or chromogenic in situ hybridization (CISH) methods for confirmation (Arnould et al. 2012). However, in the present study, FISH/CISH status of HER2 2+ cases were not directly accessible due to time constraint and so were considered as positive for ease of analysis. The assumption of HER2 2+ cases as truly positive may have caused selection bias in this study and possibly the high discordant rates seen in HER2.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	In conclusion, ER and PR immunohistochemistry performed on CNB produce results which accurately reflect those assessed on EB. The concordance rate for HER2 was less consistent, suggesting intratumoural heterogeneity among contributing factors. Concurrent analysis of HER2 with FISH/CISH on breast cancers is required for HER2 2+ cases given the high discordant rates between EB and CNB.</div>\r\n<div>\r\n	Acknowledgement</div>\r\n<div>\r\n	The authors express their thanks and gratitude to Professor Dr. Srijit Das for the help received in technical editing. This study was funded by the Medical Faculty of Universiti Kebangsaan Malaysia grant number FF-077-2013.&nbsp;</div>\r\n', 1),
(1326, 1317, '<div>\r\n	Ankle injuries are one of the most common injuries which are sustained during sports activity (Waterman et al. 2010; Doherty et al. 2014). The frequently encountered complication following ankle injury is Functional Ankle Instability (FAI), which are necessitated to be examined and intervened as it may build up to residual ankle instability and disability (Lee et al. 2006). &nbsp;FAI will cause the tissue to be damaged with proprioceptive deficits and decrease in proprioceptive feedback. Clinically it is important to regain full function and sufficient functional stability to prevent recurrent injuries and to improve the performance of an individual. Intense proprioceptive training redevelops or enhances the proprioceptive sense and feedback mechanism in athlete with FAI (Bernier &amp; Perrin 1998). The advanced equipments such as Biodex stability system, Nintendo Wii Fit and Vibrosphere TM are utilized to treat the proprioceptive deficit and balance impairment. In the absence of expensive and sophisticated equipments, the balance training can be delivered by means of utilizing simple device like wobble board. The wobble board training improves the proprioception and balance through stimulation of mechanoreceptors which are available in the muscle, tendon, ligament and joint through loading mechanism (Hubscher et al. 2010). An earlier literature also described the significant effect of wobble board training in FAI (Freeman et al. 1965). On the other hand another evidence suggests that wobble training is not promoting balance following FAI. However, the evidence is scarce on the impact of wobble board training in improving balance among function ankle instability subjects (Azeem et al. 2010). The inconsistencyof the results on the effect of wobble board training on balance promoted to carry out a study on the effect of wobble board training program on static and dynamic balance. Thus, the present study investigated the effect of wobble board training on balancing skills among athletes with FAI.</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	A quasi-experimental, pre-post design was used in this research. Thirty athletes with a self-reported FAI were selected from the department of physical education and sports science of a University. FAI is a term in which patient experiences recurrent sprain or feeling of &ldquo;giving way&rdquo; and it is a subjective and symptomatic feeling. The study protocol was approved by institutional ethics committee and written informed consent was taken from all subjects before inclusion. All the subjects were examined to meet the selection criteria as set by the study protocol. The athletes aged between 18 to 25 years old, with the history of significant episode of unilateral ankle injuries within the past twelve months were recruited for the study. The subjects were also free of existing symptoms of pain, swelling and restricted range of motion of the ankle, history of fracture, surgeries and none of them had anyform of rehabilitation program while recruitment.</div>\r\n<div>\r\n	Balance Assessment</div>\r\n<div>\r\n	The static balance was measured by single limb stance test and the dynamic balance was assessed by functional reach test.</div>\r\n<div>\r\n	Single Limb Stance Test [SLST]</div>\r\n<div>\r\n	The test was developed for testing static balance. The subjects were instructed to stand on one leg with eye closed, placing arms across the chest with hands touching the shoulders. The command was given such that both legs should not touch each other. The trial was conducted with shoes off and was about three feet from the wall. The criteria to terminate the test were the legs should not touch each other, the feet/arms moved or the feet touches down, eyes opened while performing eye closed test. The test was in accordance with the protocol proposed by (Bohannon et al. 1984).&nbsp;</div>\r\n<div>\r\n	Functional Reach Test [FRT]</div>\r\n<div>\r\n	The test was developed for testing dynamic standing balance. The individuals were subjected to stand in a relaxed standing position by making a fist and raising their dominant arm until shoulder flexion was parallel with yard stick [approximately 90 degrees of flexion]. The third metacarpal was recorded along the yardstick as position 1. Then the individuals were asked to reach as forward as they can without touching the wall and taking a step. The position of the third metacarpal along the yard stick was again recorded as position 2. The differences between the two positions were noted and this was in accordance with a proposed protocol (Duncan et al. 1990).&nbsp;</div>\r\n<div>\r\n	Wobble board training</div>\r\n<div>\r\n	The wobble board used was circular, made of wood with 430 mm diameter and height of 65 mm, which allowed 360&deg; tilt. The training protocol was adapted and modified from a previous study (McGuine &amp; Keene 2006). The athletes were asked to perform 10 min warm up which includesstretching drills and jogging. Training protocol includes five sessions in a week for four weeks; each exercise was performed for 30 sec with 30 sec rest interspersed between each exercise (Table 1). In order to acquire the skill and experience the exercise regimen, the first week of training were assisted by the physiotherapist and the athletes were instructed to perform the activity without the edge of board touching the floor. &nbsp; &nbsp;</div>\r\n<div>\r\n	Statistical analysis</div>\r\n<div>\r\n	Data was analyzed using SPSS version 17. The pre-training value and post- training value data were analyzed by using descriptive statistics of mean and standard deviation and paired t test was used to compare the difference between pre and post treatment session with in the group. Level of significance for all tests was set at p&lt; 0.005.</div>\r\n<div>\r\n	&nbsp;Results</div>\r\n<div>\r\n	A total of fifteen subjects participated in this study with a mean age of 21 &plusmn; 1.266 years old. The height and weight of the participants ranged with mean of (168.90, 59.40) and standard deviation of (8.89, 7.84). The results for single limb stance test showed that the athlete in post-training attained a high mean value (26.4), when compared to pre-training sing limb stance score (9.73). Thus, the difference was statistically confirmed by the obtained t value (16.00), which was significant at 0.05level (Table 2). The results for functional reach test showed that the athletes in post-training attained a high mean value (15.88) when compared to pre-training functional reach test score (14.26). Thus, the difference was statistically confirmed by the obtained t value (12.54) which was significant at 0.05 levels (Table 3)</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Balance training is believed to be one of the principal constituent of rehabilitating an athlete with functional ankle instability. Among the various training techniques, wobble board training is one of the cost-effective, easily administrable and challenging methods for training balance among functional ankle instability as against expensive techniques.&nbsp;</div>\r\n<div>\r\n	The present study findings suggested that wobble board training promoted both static and dynamic balance among athletes who are ailing from functional ankle instability. While comparing the improvement between static and dynamic balance, static balance values significantly improved comparatively as against dynamic balance. The reason could be the demanding task constraints imposed on the dynamic task when compared to static balance. The results of the study was also in line with an earlier study in which the authors concluded that wobble board training improved balance significantly when compared to subjects who underwent electrical stimulation and no treatment (Bernier et al, 1998). Similarly, few other studies also established that wobble board training is effective which is in line with the present study (Clark &amp; Burdern 2005; Ross &amp; Guskiewicz 2004; Hoffman &amp; Payne 1995; Mattacola &amp; Lloyd 1997).</div>\r\n<div>\r\n	However, the present study adapted different methodological perspective in terms of training as against the protocol which was used in those studies. The protocol was modified and adapted from an earlier study (Mcguine &amp; Keene 2006). The training protocol increased the intensity and reduced the duration in-order to accomplish former benefit. The predesigned methodological perspective which was adapted also proved to be an effective protocol for training functional ankle instability among athletes. The training protocol was supported by a previous work in which they adapted four weeks of wobble board training which brought significant improvement. Hence, it can be comprehended that it promoted different perception of the subjects&rsquo; functional stability and reduces further sprains in individuals with functionally unstable ankle (Clark &amp; Burdern 2005). The probable reason behind the changes in the amount of balance could be due to destabilization of load at the limb which would have induced a specific neuromuscular response. The response would also have focused on the athletes awareness of peripheral sensation and process these signals into more coordinated manner. The reason behind selecting the outcome measures such as single limb stance test and functional reach test in the present study were proved to be a simple, valid and commonly used in the evaluation and training of balance (Freeman et al. 1965). Also, it was found that all of the subjects completed each component of exercises and none of them had exhaustion effect over repeated trails.&nbsp;</div>\r\n<div>\r\n	The results of the present study demonstrates the importance of wobble board training on these special populations. Health care professionals who are working on these special populations should consider using wobble board as an additional treatment along with routine management for ankle instability to promote stability. The limitations of this study were the lack of information on the mechanism of injuries, severity of the reported ankle injuries and unavailability of control group. Therefore, it can be ordered that controlling these confounding factors would have produced suitable results. Hence, future studies need to be carried out by increasing the sample size and controlling these confounding factors to acquire appropriate results.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Wobble board training might be effective in improving balance among athletes with functionally unstable ankle. &nbsp; &nbsp;</div>\r\n<div>\r\n	Acknowledgment</div>\r\n<div>\r\n	The authors acknowledge the subjects who participated in the study.</div>\r\n', 1),
(1329, 1320, '<div>\r\n	Nasopharyngeal carcinoma (NPC) is the most radiosensitive head and neck cancer in which radiation therapy is the primary treatment modality. The incidence rate ranges between 10 and 50 per 100 000 (Ho 1978). Radiation doses between 40-70 Gy is typically delivered to adequately treat the entire tumor without damaging local structures such as the parotid gland and optic nerve (Puri et al. 2005). In a recent cohort of over 2500 patients with NPC, the control rate with these doses of radiation alone at 5 years was 85%. Although the rate increases with higher doses, greater local side effects such as radiation optic neuropathy is commonly seen (Lee et al. 2005).</div>\r\n<div>\r\n	Radiation-induced retinopathy (RR) is a delayed progressive retinopathy thought to be due to selective destruction of capillary endothelial cells leading to vascular occlusion, capillaries atrophy and subsequent ischemia (Noble &amp; Kupersmith 1984). The onset varies from 1 month to 15 years, though noticeable clinical signs usually appear within 3 years of treatment (Parsons 2004). The overall incidence of RR in patients with NPC varies with the dose of radiation (Takeda et al. 1999) with reported incidences as high as 56% with doses greater than 50 Gy and as low as 16% with lower doses (Rosenblatt et al. 2003). The dose response curve of radiation-related ophthalmopathy is sigmoidal with the risk of RR increasing steeply above 45 Gy and complete blindness reported with doses as low as 60 Gy (Parsons et al. 1983). RR is rarely seen below 30 Gy and rare cases of profound visual loss at these lower doses was found to be due to ischaemic maculopathy (Hempel &amp; Hinkelbein 1993). &nbsp;</div>\r\n<div>\r\n	Other factors for worse RR include radiation fraction size, area irradiated, concomitant chemotherapy, pre-existing vascular disorders like diabetes mellitus and increasing age (Brown et al. 1982).&nbsp;</div>\r\n<div>\r\n	The aim of the present study was to determine the prevalence of RR in patients with NPC who have completed radiotherapy in a tertiary hospital in Malaysia and to identify if a relationship exists in our population that mirrors that of published series, between the dose and occurrence of RR.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	A total of 42 NPC patients who had been exposed to fractionated external-beam irradiation between 2000 and 2004 in the Oncology Department, Universiti Kebangsaan Malaysia Medical Centre (UKMMC) were identified and considered for the study following informed consent. Ethical approval was obtained from the UKMMC Ethical Committee prior to study initiation (study code: FF-019-2006).</div>\r\n<div>\r\n	Patients were excluded if they had any of the following conditions i.e. significant media opacity obscuring adequate fundus view such as cataract, patients with known allergy to fluorescein dye, eyes with tumour invasion at the time of examination and patients who did not consent for fluorescent angiogram. Patients with diabetes mellitus (DM) were not excluded.</div>\r\n<div>\r\n	The principal investigator (NMD) examined the patients which included assessment of visual acuity, slit lamp examination particularly looking for the presence of rubeosis iridis, corneal and lens clarity, tonometry and dilated fundus examination. Coloured fundus photographs were taken before the patient was subjected to a Fundus Fluorescein Angiography (FFA). The FFA and fundus photos were graded by a secondary investigator (KHS) who was blinded to the patient&rsquo;s clinical details.&nbsp;</div>\r\n<div>\r\n	The radiation dose was estimated from the simulation and portal films and done in collaboration with the doctors and physicist from the Department of Radiotherapy and Oncology, UKMMC. When the globe lay at the border of the treatment field, the dose was estimated to be 50% of the total dose delivered. If the globe lay within the treatment field, the dose was estimated according to the graph obtained from a computerized plan. When the globe was shielded, the dose received by the retina was estimated to be between 2 to 5% of the total dose delivered.</div>\r\n<div>\r\n	The severity of RR by clinical fundoscopy was graded into normal, non-proliferative or proliferative retinopathy, depending on the presence or absence of RR and retinal neovascularization. The severity of RR by FFA was graded as normal, mild, moderate, severe, and proliferative RR based on the criteria adopted from the Branch Retinal Vein Occlusion Study (Branch Vein Occlusion Study Group 1986) (Figure 1-4). Absence of any angiographic changes was graded as normal, mild RR was defined with the presence of changes such as telangiectatic vessels, microaneurysms and cotton wool spots, without areas of capillary non-perfusion, moderate RR was defined when there is less than 5 disc diameter (DD) of area of capillary non-perfusion, severe RR was when there was greater than 5 DD of capillary non-perfusion and proliferative RR was defined as presence of leaking new vessels.&nbsp;</div>\r\n<div>\r\n	Data was analyzed using SPSS version 12.0. Chi Square test was used to determine any association between qualitative variables. ANOVA was used to compare the means of more than variables. The data on patients with DM was only used when assessing the association between concurrent medical illnesses and severity of radiation retinopathy. Results were given as mean and standard deviation.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 82 eyes from 42 patients were included in the study. There was a slight male preponderance with 24 (57.1%) males and 18 (42.9%) females with a male to female ratio of 1.3:1. The mean age was 50.3 &plusmn; 12.1 years, and ranged from 20 to 72 years. There were 26 Chinese patients (61.9%), 15 Malay patients (35.7%) and only one (2.4%) Indian patient, reflecting the acknowledged increased risk of NPC amongst the different ethnic groups.</div>\r\n<div>\r\n	Among non-diabetics, the prevalence of eyes with RR as assessed by FFA was 29.6% (21 eyes out of 71 eyes) with 18.3% (13 eyes) had mild RR, 4.2% (3 eyes) had moderate and 7% (5 eyes) had proliferative RR. The prevalence of clinically detectable RR by fundus photos was lower at 18.3 % (13 eyes), with 16.9 % (12 eyes) having non-proliferative RR and 1.4% (1 eye) with proliferative RR (Figure 5 and 6). When diabetic patients were included, the prevalence of RR was slightly higher (35.4% [29 of 82 eyes] with FFA and 25.6% [21 of 82 eyes] with clinical grading).</div>\r\n<div>\r\n	The proportion of eyes with more severe RR was significantly associated with higher doses of radiation, p &lt; 0.0001 (Figure 7). Half of all eyes receiving radiation doses less than 26 Gy had no evidence of RR by FFA, and increased levels of moderate and proliferative RR were seen in those receiving doses greater than 26Gy.</div>\r\n<div>\r\n	Three eyes in the study were totally blind with no perception of light (NPL) from radiation induced optic neuropathy. Twenty six percent of eyes experienced visual acuity of 6/18 or worse. When comparing mean visual acuity in Logmar to the severity of RR, worst RR is significantly associated with poorer visual acuity (p=0.003).&nbsp;</div>\r\n<div>\r\n	The commonest clinical feature seen in the studied eyes by clinical fundoscopy examination was hard exudation (14.3%) (Table 1). In contrast, the commonest manifestation seen on FFA was telangiectatic vessel (26.2%) (Table 2).</div>\r\n<div>\r\n	Macular oedema was more common than macular ischaemia (13.3% vs 4.9%). &nbsp;All eyes with macula ischaemia received radiation dose of more than 36 Gy. The mean radiation dose was higher in the ischaemic group (42Gy) compared to the oedema group (28Gy), although statistically insignificant (p = 0.076) (Figure 8).</div>\r\n<div>\r\n	DISCUSSION&nbsp;</div>\r\n<div>\r\n	RR is a potentially blinding condition which results from radiation delivered to the periorbital tissues for radiosensitive tumors like NPC. It is characterized by typical changes affecting the vasculature in the metabolically highly active macula, papillomacular and optic nerve area. Previous studies of RR have singled out the strongest risk factor to be the irradiated dose reaching these sensitive structures (Takeda et al. 1999; Rosenblatt et al. 2003). The present study attempted to identify the prevalence and clinical features of RR by using FFA in the local population.</div>\r\n<div>\r\n	The population of patient studied was largely Chinese concurring with previous studies on NPC (Chan et al. 2002). RR was present in about a third of our cases when FFA was used as the tool of detection. This is comparatively higher than previously reported studies where FFA was not used as the tool of detection (Takeda et al. 1999; Rosenblatt et al. 2003) .&nbsp;</div>\r\n<div>\r\n	The dominant risk factor for RR in the present study was the dose of radiation to the retina. RR occurred in eyes which has received radiation doses of as low as 3.5 Gy. Other studies reported significant risk factors which included the fraction size of radiation, increasing age and concomitant chemotherapy (Brown et al. 1982).</div>\r\n<div>\r\n	In the present study, the calculation of retinal dose is estimated from simulation and portal films which were 2 dimensional. Estimation of the distance of the posterior border of the globe to the radiation field border is important for the calculation of the retinal radiation dose. Three dimensional planning was not available to allow greater accuracy of dose estimation.</div>\r\n<div>\r\n	The irradiated dose to the retina varies as the retina is a curved planar structure that lines the part of the globe and hence different areas of the retina would receive different amount of radiation. The technique of parallel-opposed radiation is practiced at UKMMC and hence the temporal retina receives less radiation compared to the nasal retina. However, in this study we found that most changes occured around the macular. This reflects the hypothesis that the areas most affected by RR are metabolically the most active, in this case, the macula.&nbsp;</div>\r\n<div>\r\n	Clinically detectable RR was present in 25.6% of eyes as compared to 35.4% when FFA was used. This implies that if there are no changes on clinical fundoscopy, FFA may detect RR undetectable clinically such as areas of capillary non-perfusion. This would be helpful in identifying the cause of visual loss in cases where there are no detectable clinical signs. In most eyes with RR, vision was not compromised, as the pathological changes did not significantly involve the fovea or optic nerve. However, these patients may still develop proliferative RR because of extensive areas of peripheral capillary non-perfusion, which spares the fovea and hence maintains good vision. In the present study, the commonest feature of RR is telangiectatic vessels within the temporal vascular arcades. These telangiectatic vessels are considered to be the hallmark of radiation damage.</div>\r\n<div>\r\n	The second commonest feature was capillary non-perfusion, which usually affects the retinal area inferior to the inferior arcades. Because the sphenoid sinus, posterior ethmoid sinuses and posterior half of the orbit is included in the standard radiotherapy field, some part of retina may be radiated especially with clinical involvement of these structures by the tumour.</div>\r\n<div>\r\n	Patients who have retinopathy alone without maculopathy are usually asymptomatic unless they have other complications such as optic neuropathy. &nbsp;Other causes of poor vision in our study was advanced proliferative retinopathy which may still cause poor vision despite treatment with laser argon peripheral retinal photocoagulation because of &nbsp;ischaemic maculopathy. Macula oedema was found to be more common than macula ischaemia in our study. The eyes with ischaemic maculopathy had received a mean radiation dose of 42 Gy. Eyes with macular oedema on the other hand received lower radiation dose (mean 28 &plusmn; 17.00 Gy) whereas eyes without RR received a mean radiation dose of 24.2 &nbsp;&plusmn; 16.4 Gy. As the vessels supplying the macula area are largely end arteries ending at the fovea avascular zone, these vessels appear to be susceptible to radiation related endarteritis obliterans (Noble &amp; Kupersmith 1984).</div>\r\n<div>\r\n	The results of this study carries the implication that when detected early, the treatment of RR may be more effective and brings about better visual outcome rather than late detection. To date, the treatment of proliferative RR and radiation maculopathy is limited to several modalities, such as oral Pentoxyfylline (Gupta et al. 2001), pan retinal photocoagulation (Kinyoun et al. 1996) and intravitreal agents such as triamcinolone acetonide and anti-vascular endothelial growth factor (anti-VEGF). These agents may be more effective if RR is detected earlier.</div>\r\n<div>\r\n	The study may be flawed by the problems associated with a retrospective population such as incomplete data collection. Furthermore, there was a large sampling bias as many patients declined to participate in this study and therefore it is possible that only those with eye symptoms will agree to participate. Many patients were also excluded because of incomplete data as to whether there was tumour extension intracranially or to the anterior nose or sinuses. Tumors with these extensive extensions require a larger treatment field that inevitably results in a higher dose to the retina.&nbsp;</div>\r\n<div>\r\n	A prospective observational study on newly diagnosed NPC in which FFA is performed before and serially after radiation would more accurately detail the natural history of the development of RR and the aggravating factors for the development of RR.</div>\r\n<div>\r\n	Our recommendation is that patients who have had radiation therapy for NPC should have an ocular examination one year after the treatment and based on the clinical findings, be followed &ndash;up with yearly assessment if no clinical findings are noted. Should clinical findings be noted then an FFA is suggested, as the existence of neovascularisation may not be symptomatic and only identifiable by FFA.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In summary, about one third of the studied eyes showed features of RR. The dose of the radiation to the retina is strongly associated with the occurrence of radiation retinopathy. Worst vision is associated with more severe RR. However, as the initial visual acuity of these patients were not documented, the exact contribution of visual loss from RR is difficult to estimate.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to thank Universiti &nbsp;Kebangsaan &nbsp;Malaysia for the financial grant, Dr Razaien Easmail for the assistance in the analysis of data, Mr Zahari Baharom and Mr Saiful Bahari for performing the angiograms and Puan Noraini and Puan Asnizam for their help in recruitment of patients.</div>\r\n', 1),
(1332, 1323, '<div>\r\n	INTRODUCTION&nbsp;</div>\r\n<div>\r\n	The sudden development of a focal neurological deficit is namely defined as stroke. The common cause of stroke is either embolic arterial occlusion or bleeding in the brain (Internet Stroke Center 2008). Kapral et al. (2005) found that approximately 80% of all strokes were ischaemic and 20% were haemorrhagic. Moreover, stroke can cause a prolong disability among the post stroke survivors that can significantly affect their activity daily living. This is due to the result of stroke-related damage to important part of the neurological domains such as sensory and motor (Heart and Stroke Foundation of Ontario &amp; Registered Nurses Association of Ontario 2005.). According to Rosamond et al. (2008), approximately 15% to 30% of stroke survivors have permanent disability. Despite, medical and surgical treatment for stroke having improved over years, rehabilitation therapy plays a major role to regain the functional ability of stroke survivors. &nbsp;</div>\r\n<div>\r\n	The aim of rehabilitation therapy for the stroke survivors is to return them to the society with functional abilities. This could be achieved by recovery of the stroke deficits that may include deficits in cognition and functional abilities (Bendz 2003; World Health Organization 2002). On the other hand, the effect of stroke disability which causes impairments to physical and neurological that leads to handicapped individuals (Burton 2000). Therefore, the goal of rehabilitation therapy is to maximize the functional ability for stroke survivors (World Health Organization 2002). In line with this goal, many stroke survivors start their rehabilitation therapy as soon as possible after experiencing the event. &nbsp;This is to regain their mobility as soon as possible that will allow them to be safely functioning in their home environment.&nbsp;</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	The present study was conducted among consecutive stroke survivors who visited a rehabilitation clinic at one of the teaching hospital in Kuala Lumpur Malaysia. The participants were recruited from October 2009 to January 2010. Only patients with first-time stroke with CT scan showed lesions compatible with their neurological symptoms were included. Prior to the collection of data, the researchers performed a Mini Mental State Examination (MMSE) to assess their cognitive level of functioning. Those patients with severely impaired of cognitive functioning (MMSE&lt;24), severe aphasia, language problems (unable to understand the Malay, English or others language) even after being assisted by researcher or the patient&rsquo;s proxy, &nbsp;children with stroke, stroke patients with other neurological problems such as meningitis and, those with less than one month post-stroke duration were excluded from this study.</div>\r\n<div>\r\n	An explanation about the study was given to all the patients and a written consent was obtained from them. The study was approved by the Medical&nbsp;</div>\r\n<div>\r\n	Research Ethics Committee of the Universiti Kebangsaan Malaysia Medical Centre. Those patients that met &nbsp;the &nbsp; inclusion criteria &nbsp;were be asessed on their disability with Modified Barthel Index (MBI) questionaire. The MBI assessment scale was used to assess the basic functional status. This &nbsp;questionnaire consisted of ten activities of daily living. Out of ten daily living activities, eight were self-care activities which included feeding, transfer from chair to bed and back, grooming, toileting, bathing, dressing, bowel continence and bladder continence. The other two activities were mobility-related activities such as walking or propelling a wheelchair on a level surface 50 yards with or without devices or protheses, ascending and descending stairs. The MBI scale was used to define the level of functional dependency and furher it can be subcategerised into five levels of functional dependency. The additional data on their post stroke duration was revealed from cleckship.&nbsp;</div>\r\n<div>\r\n	All the data collected for this study were analyzed by using the Statistical Package for Social Sciences (SPSS) version 16.0 for Windows&reg;. &nbsp;Descriptive statistics was performed to determine &nbsp;the socio-demographic characteristics such as age, gender, ethnicity, and living arrangement and clinical characteristics like post-stroke duration and levels of functioning. The criterion for statistical significance was identified by a two&ndash;tailed probability value of p&lt; 0.05.</div>\r\n<div>\r\n	RESULTS&nbsp;</div>\r\n<div>\r\n	Socio-demographic characteristics&nbsp;</div>\r\n<div>\r\n	The mean age of 195 participants was 61 years (SD= 13.86, range = 22-87). Majority of participants were Chinese 100 (51. 3). There were 189 (96.92 %) still supported and stayed with their spouses/relatives.&nbsp;</div>\r\n<div>\r\n	Clinical characteristics</div>\r\n<div>\r\n	The participants had duration of post-stroke which range from minimum 1 month to maximum 79 months with median of 12 months.Their functional status was showed in the Table 1. &nbsp;</div>\r\n<div>\r\n	The relationships between post stroke duration with functional status&nbsp;</div>\r\n<div>\r\n	A Chi- square test for independence indicated there was significant relationship between post-stroke duration with functional status,&chi;2 (2, n=195) =6.455, p&lt;0.05, phi = 0.182. Those patients between the duration of post-stroke 13-24 months were independent (52.1) than &le; 12 months and &ge; 24 months.&nbsp;</div>\r\n<div>\r\n	DISCUSSION&nbsp;</div>\r\n<div>\r\n	In the present study, the functional status in performing their daily life activity were categorized into two categories. The participants were categorized into dependent and independent stage. The present study found that most of the participants were still fall into the group of dependent. This was due to most of them being in the duration of acute rehabilition process. This fact was similiar to an earlier study by Eriksson et al. (2008) where they found 73.1% of &nbsp;those post stroke survivals in the early phase to be still in the independent stage. They were involved in acute stage of rehabilitation and still depend to other for their daily living activities. Thus, the early stage of stroke was the common period for stroke survivors to have the deterioration of their &nbsp;functioning status.&nbsp;</div>\r\n<div>\r\n	It was found that the duration of post-stroke had an impact on functional status among &nbsp;the post- stroke survivors. In this study, the post stroke survivors of 13&ndash;24 months had better tolerence and high independency compared to those in less 12 months or more 24 months of post stroke durtaion. This study was similar to Elmstahl et al. (1996) whereby they found the post stroke survivors after one year of rehabilitation therapy had better functional status. This is due to the stroke survivors tend to recover better after one year of the rehabilitaion process. However, their functional status tend to decline &nbsp;after 24 months of post stroke duration. One of the common reasons this could happen was due to aging process of the stroke survivors and the reduced of rehabilitation therapy activity (Wolfe et al. 2002). &nbsp;</div>\r\n<div>\r\n	CONCLUSION&nbsp;</div>\r\n<div>\r\n	In conclusion, the post-stroke survivals those follow&ndash;up the rehabilitation therapy process more then one year have better functional status on their activity daily life. However, it depends on the severity of the stroke. Thus, those with mild stroke have better recovery outcome after a year of rehabilitation therapy.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1342, 1333, '<div>\r\n	Introduction</div>\r\n<div>\r\n	The metatarsals comprise five bones numbered from medial to lateral side. Each metatarsals has a distal head, shaft and a proximal base. The first metatarsal bone is large, strong and plays an important role in supporting the weight of the body. The fifth metatarsal has a prominent tubercle on its base which can be easily palpated along the lateral border of the foot. The tubercle gives attachment to the peroneus brevis tendon (Snell 2007). The metatarsals are classified as miniature or &lsquo;short&rsquo; long bones; they are peculiar in having a single epiphysis at one end only (Patake &amp; Mysorekar 1977). Typically, epiphyses appear for the base of the first metatarsal and for the heads in the remaining metatarsals (Patake &amp; Mysorekar 1977).&nbsp;</div>\r\n<div>\r\n	The metatarsals are basically supplied by the nutrient artery. Nutrient artery being the source of nutritional supply plays an important role in fracture healing and union (Shrimankar et al. 2010). The anatomical knowledge regarding nutrient artery and nutrient foramen is vital to avoid complications like gangrene and necrosis. The operating surgeon should preserve the arterial supply in free vascularized bone grafts, which is essential for the survival of osteocytes and osteoblasts (Green 1988). The knowledge about nutrient foramina is important in surgical procedures like utilization of metatarsal bones in the foot reconstruction. Singh (1960), while studying the variations in the articular facets of metatarsal bones, made some observations on their nutrient foramina. The nutrient foramina of long bones were studied by few authors (Collipal et al. 2007; Kizilkanat et al. 2007; Murlimanju et al. 2011; Prashanth et al. 2011), however the studies about the nutrient foramina in metatarsals are extremely rare. In the present study, the objectives were to observe the morphological features and location of nutrient foramen of human metatarsal bones and to figure out the index of the foramen.</div>\r\n<div>\r\n	Materials and Methods</div>\r\n<div>\r\n	The present investigation included 271 human metatarsal bones, among which 54 were first metatarsals, 53 were second, 54 were third, 57 fourth and 53 fifth metatarsal bones. The metatarsals were unpaired, the gender and age of them were not known. The metatarsals were collected from the osteology section of department of anatomy at our institution. The present study had the approval of our institutional review board. The metatarsals, which were pathological, were not included in the present investigation. The topography, number and the direction of the nutrient foramen were observed in each metacarpal bone. Only, the diaphysial nutrient foramina were studied. The nutrient foramina over the diaphysis have been described to provide the passage for the main nutrient artery and the foramina at the epiphysis were known as the vascular foramina. The vascular foramina usually transmit smaller arteries and veins. The epiphyseal foramina were not considered in the present study. The macroscopic observation in the present study was performed and recorded by the same single person. This was done to prevent the inter observer variation. The observations were done with the use of magnifying lens. A clearly seen groove which is leading into a hole and which has a raised edge at its commencement is considered as a nutrient foramen in each bone (Figure 1). The patency of the foramen is confirmed after passing a 24 gauge needle in each bone. Foramen was considered for the present study only if the 24 gauge needle enters the medullary cavity, of the metatarsal bone. The topography and number of the foramina with respect to the border and surface of the metatarsal bone was examined. The number of nutrient foramen is important, because the multiple foramina transmit the accessory nutrient arteries. Their knowledge is important to the operating surgeon to prevent the complication. The number of epiphyseal foramina was not considered in the present study, because they are not the main source of blood supply to the bone. The size of the foramen may be proportional to the diameter of the lumen of the bone. However, this was not considered in the present study. The minute arterioles which connect the Volkmann&rsquo;s and Haversian canals from the periosteum were also not considered, as it was difficult to interpret them in the dried bones. They offer a minimal quantity of blood supply to the bone.</div>\r\n<div>\r\n	The foramen index (F.I.) of each metatarsal was determined by using the formula given by Hughes (1952). As per this formula, the distance of the nutrient foramen from the proximal end of the metatarsal bone (D) was divided by the total length of the metatarsal bone (L) and the value obtained was multiplied by one hundred:</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	F.I. = D / L x 100</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The total length of metatarsal bone was determined from its base to head after using a digital vernier caliper. The F.I. was determined for all metatarsal bones, which figured out the location of nutrient foramen. The morphometric data was tabulated, each metatarsal was split into five equal parts and the location of foramen was analyzed topographically. Some of the metatarsals of the present study were radiographed (Figure 2) to observe the foramen radiologically. The films were taken using the diagnostic X-ray imaging system. The X-ray tubes were operated at approximately 60 kiloVolt peak (kVp) and 10 milliAmpere second (mAs) at a 100 cm source to image receptor distance (SID).</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	The present study observed that, among 271 metatarsal bones, 79.3% had single foramen, 9.2% had two foramina, 1.1% of had three foramina and in 10.4% of metatarsals, the foramen was not observed. The morphological distribution of the nutrient foramina of the present study was shown in Figure 3.&nbsp;</div>\r\n<div>\r\n	In the present study, 70.4% of first metatarsals had a solitary nutrient foramen. The dual foramina were found in 22.2% of cases and the triple foramina were observed in 3.7% of cases. The foramen was absent in 3.7% of first metatarsal bones. The foramina were on the lateral surface in 41 metatarsals and over the plantar ridge in 11 cases. The foramina were not observed on the medial surface of any of the first metatarsal bone. The detailed analysis of the foramina of the first metatarsal bone is represented in Table 1.</div>\r\n<div>\r\n	Analysis of the second metatarsal showed that, 83% had solitary foramen, 3.8% had dual foramina and in 13.2% of the bones, foramen was not observed. The foramen was present at the medial surface in 12 cases, at the lateral surface in 32 and over the plantar ridge in 2 second metatarsals. The frequency of distribution of second metatarsal nutrient foramen was shown in Table 2. In the third metatarsal, 83.3% had solitary foramen, 3.7% had dual foramina and in remaining 13%, the foramen was absent. It was observed at the medial surface in 29 cases, at lateral surface in 14 metatarsals and at the plantar ridge in four cases. The distribution was represented in Table 3.&nbsp;</div>\r\n<div>\r\n	The fourth metatarsal had single foramen in 77.2% of cases, double in 14% cases, triple in 1.8% cases and in 7% cases, the foramen was absent. The foramen was present at the medial surface in 31 bones, at the lateral surface in 21 fourth metatarsal bones and over the plantar ridge in 1 case. The detailed analysis of fourth metatarsal bone was shown in Table 4. In contrast, the fifth metatarsal had single foramen in 83% of cases, double foramen in 1.9% cases and in 15.1% cases, the foramen was found to be absent. It was present on the medial surface in 8 cases and was above the plantar ridge in 17 cases. The foramina were not observed on the lateral surface in the fifth metatarsal. Table 5 showed the morphological distribution of foramina of the fifth metatarsal bone.</div>\r\n<div>\r\n	The mean foramina index was 65.3 for the first metatarsal, 43.6 for second, 44.4 for the third, 45.3 for fourth and 47.5 for the fifth metatarsal bone. Most of the foramina (70.2%) in the first metatarsal were present at the 3/5th part, 57.4% of second metatarsal foramen were at the 3/5th part, 80% of third metatarsal nutrient foramina were located at 3/5th part, 88.9% of the fourth metatarsal foramen were at 3/5th part and 93.5% of the fifth metatarsal foramen were at 3/5th part from the base of each metatarsal bone. Table 6 analyzed the topography of the foramina along the length of the metatarsals, i.e. in the 1/5th, 2/5th, 3/5th, 4/5th and 5/5th parts as seen from the foraminal index. It was observed that the nutrient foramina of 2nd to 5th metatarsals were directed towards the base, whereas the first metatarsals had foramina which were directed towards head.&nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The nutrient artery being the main source of nutrition to the long bone is important especially during the childhood and puberty (Patake &amp; Mysorekar 1977). Nutrient foramen is the external opening of nutrient canal which has a particular position for each bone. The topographical information of nutrient foramen could be required in few surgeries (Malukar &amp; Joshi 2011). It is stated that the vessels, which occupy the nutrient foramen, are derived from those which took part in the invasion of ossifying cartilage, so that the nutrient foramen was at the site of original center of ossification (Payton 1934). There is slanting of the nutrient canal during the growth. The direction of slanting from the periosteal surface towards the bone marrow cavity points towards the no growing end. This is because of the higher longitudinal development at the growing end. All the typical long bones have two epiphysial ends, the upper epiphysis and the lower epiphysis. This is in contrast to the short long bones which have only one epiphysis. The directions of nutrient foramina of all long bones are always way from the growing end (Malukar &amp; Joshi 2011). It is evident that most metatarsals have one nutrient foramen located in the middle third of bone (Patake &amp; Mysorekar 1977). Singh (1960) observed double nutrient foramina occurring more commonly in the metatarsals. Also, he found that double foramina were most common in the first and fifth metatarsals. Patake and Mysorekar (1977) also reported that the double foramina were seen most frequently in the first metatarsal. The present study agrees with these reports as the double foramina were observed in 22% of the first metatarsals. In cases where double nutrient foramina are located on the same surface, it is likely that the nutrient arteries arise from the same artery. When the foramina are located on different surfaces, the source of arteries is likely to be different (Patake &amp; Mysorekar 1977).</div>\r\n<div>\r\n	&nbsp; &nbsp;The direction of the nutrient foramen is determined by the growing end of the long bone. The growing end develops at least twice as fast as the non-growing end (Mysorekar 1967). The growth rates at two ends and bone remodeling are the factors which might affect the position of nutrient foramen (Henderson 1978). It is not uncommon to see the absence of nutrient foramen in the long bone (Lutken 1950; Mysorekar 1967; Henderson 1978). After the cessation of growth, the short long bones are predominantly supplied by the periosteal vessels and the nutrient artery becomes relatively unimportant (Patake &amp; Mysorekar 1977). In cases, where the nutrient foramen is absent, it is therefore likely that the periosteal artery provides the nutrition to the bone. In the present study, 10.4% of metatarsals didn&rsquo;t exhibit the nutrient foramen. It is believed that, they are likely to be supplied by the periosteal artery. It is believed that the nutrient artery provides the major blood supply but periosteal circulation will be adequate enough for the required blood supplement. In that situation the periosteal circulation is adequate for bone healing. However, the importance of nutrient artery for the reconstruction should not be underestimated. It was described that the potential impact of vascularized bone graft from the metatarsal bones, the 2nd metatarsophalangeal joint (MTPJ), which was based on the first metatarsal artery regardless of its dorsal or plantar origin, was usually used for the clinical joint transfer. This means conventional 2nd MTPJ harvest could not preserve the nutrient artery through the foramina because most of the foramen was located at the lateral side of the 2nd metatarsals and the mean foramina index was 43.6 for the second metatarsal. From the clinical experience, there were no reports about the bone necrosis after the joint transfer. This suggests that the blood supply to the joint may be majorly from the periosteal vessels.&nbsp;</div>\r\n<div>\r\n	Patake and Mysorekar (1977) reported that, the nutrient foramina of first and second metatarsals and usually of the third and fourth, are located on their lateral surfaces, whereas that of the fifth metatarsal is on the medial surface. Patake and Mysorekar (1977) and Singh (1960) observed that, in the fourth metatarsal, foramina were more commonly observed on the medial surface. The present investigation observed that, in the first metatarsal the foramen was always present on the lateral surface. Most of the foramina were on the lateral surface in the second metatarsal bone. Whereas the third, fourth and fifth metatarsals exhibited the foramina mostly on their medial surface. This topography of the nutrient foramen of the metatarsal bone is exactly opposite to the metacarpals of the upper limb. According to a study by Premchandran et al. (2013), the foramina were more common at the medial surface in the first and second metacarpals of the upper limb. In other metacarpals, the foramina were mostly seen on the lateral surface. This change in the topography of the foramen is due to the rotation of limb buds during the development just like the other differences between the upper and lower extremities.</div>\r\n<div>\r\n	It has been reported that the topography of the nutrient artery in the fifth metatarsal bone is important while planning a corrective osteotomy for a bunionette deformity. This would allow the most proximal osteotomy possible without interrupting the nutrient arterial supply (McKeon et al. 2013). McKeon et al. (2013) described the nutrient artery and the topography of the nutrient foramen of the fifth metatarsal bone. They observed that the dorsalis pedis artery, posterior tibial artery and the peroneal artery were providing the branch which supplied the fifth metatarsal bone. The nutrient artery was arising from the fourth plantar metatarsal artery in all of their specimens. The average distance of the nutrient foramen from the base of the fifth metatarsal bone was 26.8 mm. It was opined that the osteotomies performed within the proximal 40 mm of the bone would carry the risk of interrupting the nutrient artery, which may lead to the nonunion (McKeon et al. 2013).</div>\r\n<div>\r\n	&nbsp; &nbsp;Brookes (1963) found that, in the short long bones, in later part of fetal period, more growth occurred at the end that would develop into epiphysis. Lacroix (1951) suggested that asymmetrical muscle development could modify the traction forces acting over the periosteum. The forces are capable of modifying or even reversing the direction of entry of nutrient artery into the diaphysis. Many theories have been put forward to account for the direction of nutrient canals and also on anomalously directed ones. Among them, &lsquo;periosteal slip&rsquo; theory of Schwalbe (1876) and the vascular theory of Hughes (1952) were widely accepted among the scientific community. The present investigation observed that the direction of the foramen was away from the growing end, thereby favoring the growing end theory.</div>\r\n<div>\r\n	&nbsp; &nbsp;It was described that the anatomical knowledge about nutrient foramen is useful to the operating surgeon to avoid complications like excessive hemorrhage. In the clinical set up, bony defect following the traumatic injuries like fractures, tumor resections and pseudoarthrosis, all have been reconstructed by the bone graft procedures and the preferred modality is free vascularized bone graft (Pho 1988). It has been described that, a vascular pedicle flap from the dorsal metatarsal artery is often used in the thumb reconstruction with toe transfer. During this procedure, the pedicle at the space at first web has to be identified (Henry &amp; Wei 2010). &nbsp;According to Spanio et al. (2005), 21.2 % of the patients had an asymmetric vascular pattern in the first web space. They reported that, this data cautions the surgeons about the possibility of vascular pedicle asymmetry between two feet in bilateral toe harvest procedure. They opined that the safe and straightforward toe harvest, regardless of the vascular pedicle course variations is required by the surgeon. The amputation of the foot causes disability in day-to-day life. The complete or partial removal of metatarsal bone affects the arches of the foot and balance while walking (Hashimoto et al. 2014). In order to preserve the metatarsal bone, the free flap transfers are necessary. The condition of the artery of the limb is important for the anastomosis of the artery and vein (Hashimoto et al. 2014). The plastic surgeons can improve congenital and acquired defects, such as trauma by reshaping the tissue, based on the principles of wound healing and blood supply. The flap surgeries are the best; flaps are thicker blocks of tissue that have a blood supply (Zitelli et al. 2012). &nbsp; We believe that the anatomical detail of the nutrient artery is important to the plastic surgeons while taking the grafts. The observations of the present study may be enlightening to the plastic surgeon during the bone graft procedures. However, the present study has few limitations; there is deficiency of data of the individuals who owned these metatarsal bones. The gender based comparison was not possible in the present investigation. We could not able to segregate the metatarsals into right and left sides. So the side wise comparison was not performed. The statistical comparison was not possible since the study was from the random samples. This was a mere cross- sectional study. Correlation of data obtained in the present investigation to the physical condition of foot was not clarified.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	We believe that the present investigation has added extra information about the morphology and location of the nutrient foramen of the metatarsal bones and their foraminal index. The present study observed that metatarsal bones had one nutrient foramen which was situated in the 3/5th part of length of the bone. Few metatarsals had no foramina and some of them had double and even triple foramina. In the first metatarsal, the foramina were always present on the lateral surface. Most of the foramina were situated on the lateral surface in the second metatarsal bone. Whereas the third, fourth and fifth metatarsals exhibited the foramina mostly on their medial surface. All the metatarsal bones followed the growing end theory of the nutrient artery. The present study observed that the site of entry of the nutrient artery to the metatarsal bones had significant variations. This may suggest that the periosteal circulation is also an essential factor and this has to be considered during the reconstruction procedures.</div>\r\n<div>\r\n	Acknowledgements</div>\r\n<div>\r\n	The authors sincerely thank staff and members of the Department of Anatomy laboratory for the help given during this present study.</div>\r\n', 1),
(1344, 1335, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Adolescent has long been defined as a transition stage, from early puberty to adult, occurring between the age of 13 to 19 years. In this stage, adolescent will go through some changes such as seeking for self-identification, and forming intimation in social relationship and preparing for career. Depression is a condition where someone feels despair and emotionally pressured. According to World Health Organization, depression is a feeling disorder where it can become worst and lead to suicide or severe mental illness (WHO 2010).&nbsp;</div>\r\n<div>\r\n	Adolescents should be able to adapt with these changes, and if they have difficulty adapting, they will face some conflict that may lead to stress, anxiety and depression as well. Almost half of adolescents have difficulty overcoming stressful situation such as problems with parents, competition with peers to achieve good scores or grades and financial problem. The aforementioned difficulties may lead to depression if they fail to deal with it properly (Jas Laile Suzana 1996). Depression comprises three stages which are mild, moderate and severe depression (Beck 1967).</div>\r\n<div>\r\n	According to World Health Organization, depression is one of the most debilitating disease of the world (WHO 2010). Depression has been reported to be high in prevalence among Malaysian population, especially among women (Malaysian Psychiatric Association 2010). This causes great concern to the healthcare practitioners and policy makers (Malaysian Psychiatric Association 2010). A large scale cross sectional study in Malaysia reported the prevalence of possible depression among secondary school students was 10.3% (Ramli et al. 2008). However, most of the depression cases were not diagnosed accurately, and the sufferer did not receive proper treatment from the doctors or psychiatrist.&nbsp;</div>\r\n<div>\r\n	Depression among adolescents is one of controversial issues for the few past decades (Kauffman 2001). Normally, depression is related to few factors such as inherited genetic and sex. There are conflicting findings regarding association of sex with depression &nbsp;but studies showed females tend to be more depressed compared to males (Bachanas &amp; Kaslow 2001; Nolen-Hoeksema 2001; NIMH 2011). Adolescents from low socio-economic status may have depression throughout their transition period from adolescents to adults stage (Melchior et al. 2010). Conflict with parents are also related to depression with increasing age during adolescence stage (Lohman &amp; Jarvis 2000) and adolescents with parents&rsquo; divorce were perceived to be aggresive and possess risky behaviour (Plunkett et al. 2000). This leads to stress towards the whole family which can interupt the equilibrium of the family system (Jaycox et al. 2009).&nbsp;</div>\r\n<div>\r\n	Studies revealed that high depression in adolescence was also associated with poor peer relationships (Matos et al. 2003), having low self esteem (Rice 2000) and having low academic achievement (Lazarus 2000). The present research was carried out to determine the prevalence of depression and its predictive factors so that the early intervention could be implemented in order to reduce another mental health problem. In addition, the results of the present research may assist the parents who have adolescents with depression.</div>\r\n<div>\r\n	Materials and Methods</div>\r\n<div>\r\n	Sample</div>\r\n<div>\r\n	A cross sectional study was conducted to identify predictive factors associated with depression among adolescent from five secondary schools in the state of Selangor, Malaysia from January to Mac 2011. They were selected through simple random sampling of the name list provided by the teachers. Ethical approval was obtained from Research and Ethics Committee of Faculty of Medicine UKM (FF-009-2012), Ministry of Education, Director of Selangor Education Department and written consent from parents/guardians. In reference to a local study (Ramli et al. 2008) and using a formula (Kish 1965), a total of 160 Form Four students were required for this study. After considering missing data or non response, 20% were added giving total sample size of 191 respondents with 95% confidence and 80% statistical power.</div>\r\n<div>\r\n	Study tools</div>\r\n<div>\r\n	A set questionnaires consisting of five sections was pre-tested then distributed to selected adolescents. The questionnaire included the following sections: (A) respondent&rsquo;s demographic (i.e. gender and family income), (B) a 19 items questionnaire about &nbsp;the level of depression, (C) a 12 items questionnaire about family relationship, (D) a 10 items questionnaire about peer support, (E) a 10 items questionnaire about the level of self-esteem, and (F) academic achievement.</div>\r\n<div>\r\n	The questionnaire regarding the level of depression was constructed based from literature reviews such as Beck Depression Inventory (BDI)-Malay and Child Depression Inventory (CDI)-Malay and it was designed to meet the approval of the Ministry of Education Malaysia (Mukhtar &amp; Oei 2008; Rosliwati et al. 2008). It consisted of 9 positive items and 10 negative items. This questionnaire was used to assess the severity of depression among adolescents. Students were required to rate how often they experienced with such a situation on each statement. Each item was scored as 0 (never), 1 (seldom), 2 (sometimes) and 3 (all the times). The cut-off point was 30. A score of higher than 30 indicates the presence of depression and a score of 30 and less indicates otherwise. The questionnaire was pretested giving Cronbach&rsquo;s alpha of 0.85.</div>\r\n<div>\r\n	A family relationship questionnaire which consisted of 12 items required the students to rate each item as 0 (not at all), 1 (not much), 2 (moderate) and 3 (most of the times) was designed to suit the target population. The total score was calculated to determine the relationship between adolescents and their family. A total score of more than 10 indicated that they had a poor relationship with their family and vice versa. This questionnaire was pretested giving Cronbach&rsquo;s alpha of 0.79.</div>\r\n<div>\r\n	A questionnaire about peer support consisting of 10 items, and coefficient of 0.83 was administered. This questionnaire required respondents to rate how often they have problems with their peers. Each item was scored as 0 (no), 1 (seldom), 2 (sometimes), 3 (often) and 4 (all the times). Total score was calculated to determine the problem between adolescents with their peers. Total score of over 10 indicated that they had a relationship problem with their peers and a total score of 10 and less, indicated otherwise.</div>\r\n<div>\r\n	Self-esteem was assessed using Rosenberg Self-Esteem Scale (Mohd Jamil 2006). This scale consisted of 5 positive items and 5 negative items. It require respondents to rate how much they strongly agree and strongly disagree with each statement. Each item was scored as follows: 0 (strongly disagree), 1 (disagree), 2 (agree) and 3 (strongly agree). The minimum possible score is 0 and the maximum score is 30. Score of more than 20 indicated that an adolescent had a high self-esteem. This questionnaire was pretested giving Cronbach&rsquo;s alpha of 0.73.&nbsp;</div>\r\n<div>\r\n	For academic achievement, it was assessed using the Lower Secondary Assessment results. Lower Secondary Assessment is a Malaysian public examination taken by all &lsquo;Form Three&rsquo; students in both government and private schools, throughout the country. Grades form each 8 subjects [Bahasa Melayu (Malay Language), English, Science, Mathematics, History, Geography, Islamic Education and Living Skills] were rated from 0 to 4 (A= 4, B= 3, C= 2, D= 1 and E= 0). The rates were then &nbsp;transformed to the level of academic achievement as in Table 1.</div>\r\n<div>\r\n	Statistical analysis</div>\r\n<div>\r\n	Median and interquartile range (IQR) was used to describe the characteristics of the study population for continuous data, whereas frequency and percentage were used for categorical data. Simple logistic and backward stepwise multiple logistic regression were used to determine the predictors towards depression. The data was analysed by using Statistical Package for Social Science (SPSS) version 13.0 with the significant level was at p &lt; 0.05.</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	A total of 174 questionnaires were returned to the researchers, out of the 191 questionnaires that were sent out. All questionnaires returned were from Malay and Muslim adolescents only. Table 2 showed that majority of the adolescents were females from family with median income of MYR2,000 (1,300-3,500). The majority of them reported having poor family relationship and peer support. However, most of the adolescents had high self-esteem which maybe be reflected by their school achievement. The prevalence of depression was 50.3%.</div>\r\n<div>\r\n	Table 3 indicated there are more female adolescent, adolescents with poor family support, poor peer support and low self esteem experienced depression. Majority of the adolescents with low to moderate academic achievements encountered depression. Further analysis indicated that poor peer support as a predictive factor towards depression among adolescents in which slightly attenuated with multivariable analysis (Table 4).</div>\r\n<div>\r\n	Discussion&nbsp;</div>\r\n<div>\r\n	The aim of the present study was to investigate the effect of common factors to prevalence of depression specifically among adolescents in the urban area. The schools where the students were selected, were located in urban areas where the environment could influence the level of depression among adolescents. The present study indicated that half of students were depressed and this figure was higher compared to a study conducted in Malaysia that showed a prevalence of 24.2% (Yaacob et al. 2009). This is probably was due to the larger sample size and involved students aged 13-17 years compared to the present study which focused on subgroup of &lsquo;Form Four&rsquo; students.</div>\r\n<div>\r\n	Results from the present study showed that majority of adolescents had poor peer relationship. &nbsp;This finding is supported by many studies conducted in other countries (Anda et al. 2000; Zgambo et al. 2012). This is probably because, during adolescence, sources of attachment may come through non familial relationships at school such as with peers and friends (Kubik et al. 2003). Furthermore, as adolescents grow, they express a clear preference for spending time with peers and school or campus is a major living environment for adolescents which may influence adolescents&rsquo; mental health (Garnefski 2009). Moreover, adolescents normally discuss their romantic and sexual relations with their peer group (Wisnieki et al. 2013). Nevertheless, if adolescent has a strong peer relationship, it may be a protective factor against depression (National Research Council and Institute of Medicine 2009).&nbsp;</div>\r\n<div>\r\n	Although, it did not reach any statistical significance, majority of adolescents in the present study also reported having a poor family relationship. Many adolescents consider their inter-family communication to be poor, especially with their father and feel that their parents do not understand them or listen to their feelings (ENJ, 2000). The small sample size of the present study as compared to previous study probably lead to this insignificant finding (Adelusi &amp; Coker 2013)</div>\r\n<div>\r\n	Results from the present &nbsp;study also showed that gender was not significantly associated with depression. This result was consistent with many previous studies among adolescents (Masten et al. 2003; Sancakoglu &amp; Sayar 2012). In contrast, few past studies found that there were sex differences in depression among adolescent especially among female adolescents. The differences between sex and depression may be caused by sex role differentiation between male and female adolescents (Feldman 2007). The changes in biological, cognitive functioning and social role secondary to puberty may explain the sex differences in the prevalence of depression (Hankin 2006). The lack of sex differences in depression in the present study may also be due to the fewer number of males compared to the females.</div>\r\n<div>\r\n	Both self esteem and academic achievement did not show any significant association for this present study. However, a study showed that high self esteem can prevent depression among adolescent as it can help them to sustain good mental health and psychological state (Solberg et al. 1998). According to scar model, self-esteem is a consequence rather than a cause of depression where depression is assumed to persistently deteriorate self-esteem (Orth &amp; Robins 2013) and may leave permanent scars in the self-concept of the individual (Shahar &amp; Davidson 2003). Academic achievement is not the criterion that might influence adolescents&rsquo; self-evaluation of their own competence (Pederson et al. 2005). However, a study among Egyptian secondary school female students showed that depression was associated with poor academic performance (El-Missiry et al. 2012) as this could be due to the inability of some adolescents to cope with such situation (Da Costa &amp; Mash 2008).&nbsp;</div>\r\n<div>\r\n	This study was primarily limited by sampling of the study population, which rendered the non-generalization of the results to the general population. The study population only focused on Form 4 students who represented only 13% of adolescents in Gombak. It would be better if all students from secondary schools in that district took part in this study. Furthermore, due to the cross-sectional nature of the study design, causal inference cannot be made. Despite the limitations, this study was also with some strength whereby the questionnaire designed maybe further used as an indicator for severity of depression and not as a diagnostics tool. Future researches should look into other factors that may contribute to increase severity of depression such as the relationship between students and school environment and pressure from teachers. In addition, the division in terms of ethnic and religious factors also needs to be balanced because it will affect the result.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Results indicated that half of the adolescents in this study population experienced depression. Beside the adolescents with depression, special attention should also be given to the adolescents without depression. It is because adolescence is a stage for them to struggle with the transition process from childhood to adulthood that is accompanied by biological and psychological transformations. This is also the time for them to define their place in the family, peer groups and larger community. Therefore, prevention programs that utilizing cognitive behavioral and/or interpersonal approaches, peer support and family-based prevention strategies are possibly helpful.</div>\r\n<div>\r\n	Acknowledgement</div>\r\n<div>\r\n	The authors wish to acknowledge the financial support and approval obtained from Medical Research Committee, Universiti Kebangsaan Malaysia Medical Centre (FF-009-2012). The authors also would like to thank the Ministry of Education for granting the permission to conduct this study at five secondary schools in the district of Gombak and the parents and guardians for providing the consent for these students. The authors also acknowledge the contribution received from the school teachers and students.</div>\r\n', 1),
(1349, 1340, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Pain is defined by the American Pain Society Quality of Care Committee, as an unpleasant sensory and emotional experience arising from actual or potential tissue damage or described in terms of such damage (Lin et al. 2005). Post-operative pain is the most regular and expected complication following orthopaedic surgery (Caumo et al. 2001). Acute pain is associated with post-operative surgery; different patients even though undergoing the same incision, may perceive pain differently (Polomano et al. 2008). Nurses play an important role and responsibility in ensuring adequate pain assessment and management towards patients while being hospitalized. An adequate knowledge, beliefs and attitudes by nurses could alleviate their misconception of pain assessment and pain management (Dihle et al. 2006).&nbsp;</div>\r\n<div>\r\n	In Universiti Kebangsaan Malaysia Medical Centre (UKMMC), researchers found that many patients would rather tolerate their pain then to report to nurse for their analgesic administration. They perceived that analgesic agents should only be taken when necessary. Hence, most patients deemed to withstand their pain, resulted in poor recovery, increased length of stay, increased cost incurred and immobilization during hospitalization (Ho et al. 2013). According McCaffery and Ferrell (1997), pain is a complex physiological and psychological phenomenon which is very subjective in nature. &nbsp;Inadequate information leads to patients&rsquo; misconception and reluctance to report pain and should be overcome to ensure an effective pain management.&nbsp;</div>\r\n<div>\r\n	A typical belief among patients is that their desire to be &ldquo;good&rdquo; patients and fear of addiction with opioids could be the contributing factors to poor pain management. They are reluctant to report their pain to the physicians and nurses. A previous study reported the patient&rsquo;s belief that pain builds character as they view pain as having moral value and may lose their face values (Ho et al. 2009). Patient&rsquo;s ignorance and lack of proper knowledge related to pain management may further increase barrier to obtain and optimize pain control (Ortiz et al. 2015).</div>\r\n<div>\r\n	Post-operative pain after orthopaedic surgery can be devastating. Hence, proper pain management education package is expected to enhance optimum delivery of care in order to achieve adequate patient&rsquo;s satisfaction and comfort. The aim of the present study was to examine the impact of an education pain package and to measure to what extent it has on pre-education and post-education of orthopaedic patients&rsquo; belief towards pain management.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Design</div>\r\n<div>\r\n	This study was conducted in the Orthopaedic Surgery Wards which comprise the male Orthopaedic Ward, the female Orthopaedic Ward, Spinal Ward and Trauma Ward of UKMMC. UKMMC is a tertiary hospital which receives both emergency and referred cases. A pre-test-post-test study design was done to examine the impact of the pain education on the outcome of patients undergoing orthopaedic surgery in a tertiary hospital. &nbsp;</div>\r\n<div>\r\n	Participants</div>\r\n<div>\r\n	The recruitment of respondents in this study began when the patient was admitted to the Orthopaedic Ward for an elective orthopaedic surgery. The respondents were selected through convenience sampling. The purpose of this study was delineated by the researchers verbally to the respondents and a written consent was acquired. Prior to the routine pre-operative teaching by the ward nurse, the respondents were asked to answer the Barrier Questionaire-13 (BQ-13) as a pre-test to get a baseline data of their belief about pain and pain management.</div>\r\n<div>\r\n	Pre-operative Education Intervention&nbsp;</div>\r\n<div>\r\n	The researchers provided pain education package with routine information to the patients. Other healthcare professionals also gave intermittent usual routine care and information in terms of their surgery and anaesthesia, wound care, physiotherapy, routine hemodynamic observations and pain management. After the pre-test, all of the participants were administered a self-devised Pain Education Package which included the following: definition of pain, basic pathophysiology of pain and its effects on pain, general understanding about pain, management, medications and side effects and non pharmacological management of pain. The investigators administered the pain education package verbally with the aid of a flip chart. Estimated time of presentation was at least 15 minutes. A shorter education time was preferred to make sure that the participant was focused during the verbal administration</div>\r\n<div>\r\n	Data collection and Instruments</div>\r\n<div>\r\n	The research instrument consisted of socio-demographic data and a modified BQ-13 which was adapted from Boyd-Seale et al. (2010) with permission. A set of BQ-13 was used to assess the patient&rsquo;s pain belief, pain management and side effects of medication. Each item was measured with a 6 point Likert scale, with 0 being &lsquo;Do not agree at all&rsquo; and 5 being &lsquo;Agree very much&rsquo;. Post-test BQ-13 was administered on 3rd post-operation day.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Ethical approval</div>\r\n<div>\r\n	The research was approved by the Ethics Committee of Universiti Kebangsaan Malaysia Medical Centre (UKMMC) (Code No.FF-2014-185). Permission was granted by the Dean cum Director of Universiti Kebangsaan Malaysia Medical Centre. Written informed consent was obtained from the respective respondents. The privacy and confidentiality of each individual&rsquo; information was maintained and the subject were given the right to withdraw from participation.</div>\r\n<div>\r\n	Data analsyis techniques</div>\r\n<div>\r\n	Data analysis for this study was done using SPSS version 21. The relationship between sociodemographic variables with the BQ-13of orthopaedic surgery patients &nbsp;were analyzed by inferential statistic analysis using student t-test and Analysis of Variance (ANOVA).&nbsp;</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	The sociodemographic data consisted of gender, ethnicity, education level and history of prior surgery. There were equal amount of respondents for each gender, &nbsp;15 male respondents (50%) and 15 female respondents (50%). Majority of the respondents were Malays 21 (70%), Chinese 7(23.3%) and Indians 2(6.7%). Regarding the educational level of the respondents, 6 had primary level (20%), &nbsp;13 had secondary level (43.3%) while 11 had tertiary &nbsp;(36.7%). Seventeen respondents had history of prior surgery (56.7%) while 13 respondents (43.3%) did not have it (Table 1).</div>\r\n<div>\r\n	Table 2 reported apaired samples t-test used to comparethe Barrier Questionnaire-13 of the pre-test (Mean = 41.87, standard deviaton = 11.467) and post-test (Mean = 34.80, Standard Deviation = 13.026). There was a reduction of &nbsp;mean score of pain belief of 7.07. This was statistically significant (t = 2.84, p = 0.004). The pre-test of pain management (Mean = 27.47, Standard Deviation = 6.857) and post-intervention scores (Mean = 22.03, Standard Deviation = 8.393) was statistically significant (t (9) = 3.58, p = 0.001). Pre-test and post-test of side effects of pain medication &nbsp;reported a mean score and standard deviation of (Mean = 14.47, Standard Deviation = 6.806) and (Mean = 12.77, Standard Deviation = 6.339) respectively. However, this difference was statistically insignificant, t (29) = 1.257, p = 0.219.</div>\r\n<div>\r\n	There were statistically significant differences between gender with BQ-13 where the male respondents &nbsp;(Mean = 37.13, Standard Deviation = 9.790) scored higher than female (Mean = 46.47, Standard Deviation = 11.420 ); (t &nbsp;= -2.403, p = 0.023). There was no significant difference in terms of history of prior surgery (p=0.289) as shown in Table 3. In Table 4, there were no significant differences between respondent&rsquo;s pain belief with ethnicity and education level respectively (p=0.426 and p= 0.361).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Pain is a complex physiological and psychological phenomenon which is very subjective in nature (McCaffery &amp; Ferrell 1997). Inadequate information leads to patients&rsquo; misconception and reluctance to report pain and should be overcome to ensure an effective pain management. A typical belief among patient is their desire to be a &ldquo;good&rdquo; patient and fear of addiction with opioids could be the contributing factors. In Malaysia, the pain management barriers are caused by the physicians&rsquo; and patients&rsquo; attitudes towards the use of opioids (Lim 2008). Patients possess many different perceptions related to pain and its management. Failure of healthcare providers to assess and document pain has been one of the major barriers for an effective pain control.</div>\r\n<div>\r\n	The findings from this study indicated &nbsp;reduction between &nbsp;pre-test and post-test scores of pain belief and management among patients undergoing orthopaedic surgery. Conclusively, the decrease in the pain belief and management scores were statistically significant which implicate the effectiveness ofpain education package in this study. In a previous study by Taylor &amp; Stanbury (2009), it was shown that pain information provided by health care providers would &nbsp;enhance patients and family members of their awareness towards pain management. The provision of information and appropriate education tailored to the patients&rsquo; needs and level of understanding would further compliment in reducing patient anxiety and avoiding unrealistic expectations related to the pain relief (Al Samaraee et al. 2010).</div>\r\n<div>\r\n	There were no significant differences between the pre-test and post-test scores for side effects of pain medication. They are reluctant to report their pain to physicians and nurses. According to Ho et al. (2009) patients believe that pain builds character as they view pain as having moral value and may lose their face values. Patient&rsquo;s ignorance and lack of proper knowledge related to pain management will further increase barrier to obtain optimal pain control. They would rather withstand the pain than to get the pain relieved. Patients dread the side effects of the opioid analgesics, and this may be supplemented by the bad attitude of the nurses in administering opioid analgesics to patients (McCaffrey &amp; Ferrell 1997; Taylor, &amp; Stanbury 2009). Another study reported a high proportion of patients who believed that usage of analgesics may cause addiction to pain medication easily (Bozimowski 2012). The information regarding analgesic in pain management is important in pain relief towards pain belief after six hour surgery (Brown et al. 2013). This showed that effective information delivered may give positive improvement in patient&rsquo;s pain belief towards pain management.</div>\r\n<div>\r\n	Female patients scored higher for pain belief compared to males. This was probably because women were more open to pain and were willing to share with others regarding their pain compared to males who are more reserved and would not divulge their problem for fear of losing face (Patel et al. 2014). Also, there was no significant difference in terms of prior surgical history although those without prior surgical history scored higher in terms of pain belief. This could be due to those who did not experience surgery beforehand, and had higher acceptance of analgesics compared to those who had experienced surgery and who may have a higher threshold of pain.&nbsp;</div>\r\n<div>\r\n	There were no significant differences in pain belief between ethnicities although Chinese scored the highest while Indians were the lowest. This could be due to Indians having lower threshold of pain though this is not proven as yet.In contrast, Kapoor and Thorn (2014) showed that ethnicity did not influence the healthcare use or prescription of opioids. &nbsp;In terms of education, tertiary education had the highest score with the lowest being primary. This is expected as the higher the education level, the better the understanding of pain relief management. According to Costello and Thompson (2014), knowledge gap and nurses&rsquo; lack of sufficient information about opioids may affect their ability to provide effective medication instructions to their patients</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, there was a positive impact of the pain education package towards pain belief, management and side effect of medications. Reinforcement of pain educational program is pivotal in order to achieve optimal post-operative pain management. Patients will achieve better comfort, faster mobilisation and reduced length of stay. This may be cost effective and time saving for both patients and the organisation.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(1352, 1343, '<div>\r\n	Introduction</div>\r\n<div>\r\n	According to the American Academy of Orthopedic Surgeons (AAOS), 32.9 million people had been diagnosed with some types of arthritis, in the United States (Prouty et al. 2006) and about 250,000 knee replacement surgeries were performed annually (Dahlen et al. 2006). Pain after surgery can be considered a form of acute pain with inflammatory responses resulting from surgical tissue damage. It is estimated that approximately 80% of patients experience post-operative pain, of which 86% have moderate, severe, or extreme pain (Elliott 2011).</div>\r\n<div>\r\n	Adequate pain control assists in early post-operative mobility, earlier hospital discharge, increased patient satisfaction, reduced recovery time (Herkowitz et al. 2007; Simcock et al. 2008). Patients will experience less pain, reduced physiological and psychological problems (Gillaspie 2010). Pain can reduce range of motion in knee joint, cause limitation in leg movement, muscle weakness, muscle wasting (Dahlen et al. 2006), delayed healing, reduced immune function, decreased quality of life, increased stress and anxiety. It may also cause increased re-admission rates, increased healthcare costs (Gordon et al. 2000), increased analgesic usage, and also symptoms like nausea and vomiting (Indelli et al. 2005).</div>\r\n<div>\r\n	Post-operative pain in adults is usually not adequately managed because nurses fear addiction and complications of analgesic (McCaffrey &amp; Locsin 2006). Therefore, combination of analgesic with non-pharmacological intervention may become an effective measure to reduce post-operative pain. Music therapy is one method of non-pharmacological intervention that can reduce post-operative pain (Masuda et al. 2005). Listening to music is a noninvasive, safe and inexpensive nursing intervention that has been successfully used in hospitals (McCaffrey &amp; Locsin 2006). The aim of the present study was to identify the effect of music therapy on pain after TKR surgery and usage of analgesic among patients admitted in UKMMC.</div>\r\n<div>\r\n	The effect of non-pharmacological intervention on post-operative pain was practiced after various elective surgeries such as orthopedic, abdominal, cardiac and gynecology. A few of them found significant difference in pain score between the music therapy group and the control group (Ebneshahidi &amp; Mohseni 2008; Good et al. 2005; Hook et al. 2009; Tse et al. 2005) and the others figured out that there was no significant difference in pain score between the music therapy group and the control group (Allred et al. 2010; Pellino et al. 2005). Few studies have reported that music therapy can reduce usage of post-operative analgesic (McCaffrey &amp; Locsin 2006; Ebneshahidi &amp; Mohseni 2008; Tse et al. 2005; Pellino et al. 2005). Intra operative music therapies had also been performed and it was mentioned that music therapy could reduce post-operative pain (Simcock et al.2008; Mottahedyan et al. 2012) and also minimize excessive usage of analgesic (Mottahedyan et al. 2012).</div>\r\n<div>\r\n	Research in music therapy has revealed that it not only reduces pain but also helps in overall physical rehabilitation, facilitates ambulation, motivates patients to become engaged in their treatment and also provides emotional peace to patients (American Music Therapy Association Inc). Gallagher (2012) explained that the effect of music on pain relates to the gate control theory, that when a patient is focused on the music, this is what goes through the neurotransmitters at the time and it shuts the gate on pain because its hard for a person to focus on two things at one time.&nbsp;</div>\r\n<div>\r\n	The aim of the present study was to determine the different levels of post-operative pain score between the group using music therapy and the control group. We also aimed to determine the usage of analgesics between the experimental and control group.</div>\r\n<div>\r\n	Materials and Methods</div>\r\n<div>\r\n	McGill Pain Questionnaire</div>\r\n<div>\r\n	Pain was measured by short form of the McGill Pain Questionnaire (MPQ-SF) that consisted of three parts: A) Pain Rating Intensity (PRI): 15 descriptors of pain (11 sensory; 4 affective) which were rated an intensity scale as 0 = none, 1 = mild, 2 = moderate, and 3 = severe, three pain scores were derived from the sum of the intensity rank values of the words chosen for sensory, affective and total descriptors. B) Visual Analogue Scale (VAS) (0-10). C) Present Pain Intensity (PPI) which were rated on intensity scale as No Pain = 0, Mild = 1, Discomforting = 2, Distressing = 3, Horrible = 4, Excruciating = 5 (0-5) (Melzack 1987). Permission was requested from Dr. Melzack and a pilot study was carried out for the Malay version of the questionnaire, the Cronbach&rsquo;s Alpha was 0.876. Reliability and validity of MPQ-SF had been proved in many studies in countries such as England, Thailand, Turkey etc (Melzack 1987; Kitisomprayoonkul et al. 2006; Yakut et al. 2007).</div>\r\n<div>\r\n	Music Set</div>\r\n<div>\r\n	The music set for this study consisted of music booklet, compact discs (CD) bag consist of 14 CD, head phone, and DVD player. The first option to listen to music was patient&rsquo;s choice. Patient could bring the CD from their home to the hospital or they could choose their favorite CD from the researcher&rsquo;s collection (Simcock et al. 2008; McCaffrey &amp; Locsin 2006; Heitz et al. 1992). Collection of music for this study was based on soothing and relaxing music (Good et al. 2005; Tse et al. 2005; Bunt &amp; Stige 2014; Shertzer &amp; Keck 2001). This researcher&rsquo;s collection consisted of 6 types of music which were soothing and relaxing music without lyric: 1) Relaxing and soothing music, 2) the nature sound (meditation), 3) soul relaxation, 4) romantic piano, 5) classic collection and 6) violin music. Lo (2012) reviewed ten articles using music therapy for reducing pain after TKR surgery. They recommended guidelines that consisted of sedative or relaxation music (music which had no lyrics), sustained melodic quality; absence of strong rhythms or percussion; rate of 60-80 beats per minutes). In this study, the patients were instructed to listen to music for an hour, four times in a day depending on their choice of time for listening (McCaffrey &amp; Locsin 2006).</div>\r\n<div>\r\n	Study Design</div>\r\n<div>\r\n	A quasi-experimental design (non-equivalent post-test-only control group) was used for this study. A convenience sample of 58 patients was obtained from the Orthopedic Ward of UKMMC over a period of 8 months (from May 2012 to December 2012). Sample size was calculated by PS version 3.0.4 by using research of Simcock et al. (2008), power 0.80, alpha=0.05, and in dependence T-test. The sample size was 40 (experimental group: 20 and control group: 20). The inclusion criteria were: patients aged 40-80 years-old, scheduled for elective TKR, able to listen to music, able to communicate in English or Malay language, able to fill out the questionnaires, alert and oriented to time, person and place. The exclusion criteria consisted of history of mental illness, allergic to analgesics, patients admitted to intensive care unit (ICU) post-operatively, hemodynamically unstable and patient dependent on opioids.</div>\r\n<div>\r\n	Data collection</div>\r\n<div>\r\n	The study was approved by the UKMMC Ethical committee on 4th April 2012. Each patient was informed on the importance of study one day before surgery when admitted to the ward. Patients who agreed to participate were asked to fill up the consent form and return it to the researcher. There were 58 patients who willingly participated in the study but 8 of them were excluded due to various reasons (post-operative ICU admission, discharged earlier, addiction, hearing problem, age less than 40, communication problem) and 10 patients refused to participate in this study (Figure 1). Demographic data was obtained and McGill questionnaire was explained to the patients for both groups.&nbsp;</div>\r\n<div>\r\n	To ensure the smooth flow of the process, there were certain rules the patients had to follow. They were not allowed to bring electronic devices such as radio, CD player and laptop. All other instructions were the same for both groups including surgeon, operation room, anesthesia medication, private room post-operatively, type of pain relief medication. The patients were divided into two groups with 20 in each group using the sealed envelope technique. The two groups in this study were the experimental group who received music therapy post-operative to TKR, control group who did not undergo any intervention. Both groups received pain relief as ordered by the doctor.</div>\r\n<div>\r\n	In the experimental group, the musical set was given to the patient a day prior to surgery (Day 0) and the patient kept the music set till discharge from the ward. The experimental group listened to music after surgery for an hour, four times a day. The two groups were monitored by the researcher and nurses, the questionnaire was taken on post-operative day 1, post-operative day 3 and post-operative day 5. The time for filling the questionnaire was the same for all patients. Patients were still in the rest position before taking medication at noon. The dosage of analgesia used was measured and calculated over five days post-operatively and compared between two groups. The majority of medication used for post-operative pain control was Paracetamol, Celecoxib and Tramadol.</div>\r\n<div>\r\n	Data analysis</div>\r\n<div>\r\n	All collected data were converted, coded and transferred into the SPSS program version 19 to carry out an analysis. Data was not normally distributed after conducting Skewness, Kolmogorov-smirnov, Shapiro-Wilk and histogram normality test. The researcher therefore used median and mean rank for analyzing the data Mann-Whitney test was used between groups&rsquo; comparison and Friedman test was used within group comparison for pain score. Mann-Whitney test used dosage of analgesics between two groups.</div>\r\n<div>\r\n	Results&nbsp;</div>\r\n<div>\r\n	Socio-Demographic Data of Patients</div>\r\n<div>\r\n	There were 32 (80%) females and the rest were males. The mean age of patients who participated in this study was 64.35 years old (49 to 76 years). The racial distribution was 20 Malays (50%), 3 Indian (7.5%) and 17 Chinese (42.5%). Most of the patients were married 36 (90%). The religion of patient were: 20 Muslims (50%), 3 Hindus (7.5%) and 17 Buddhist (42.5%). Equal percentage of patients had primary school education and secondary education (47.5%) where only 2 (5.0%) patients had tertiary education, and majority of patients were unemployed 39 (97.5%). There were significant statistical differences in terms of race and religion and the comparison between the two groups is shown in Table 1 and 2.</div>\r\n<div>\r\n	Analysis of PRI between Two Groups on Day 1, 3 and 5</div>\r\n<div>\r\n	The Pain Rating Intensity (PRI) was not normally distributed for analysis of pain score between the two groups using Mann&ndash;Whitney test, the results showed that there was significant difference in pain score between experimental and control group at 0.05 level on day 1(P = 0.00), day 3(P = 0.00) and day 5 (P = 0.00) post-operatively using PRI. Therefore, with the above results and compared mean rank between two groups, it could be concluded that pain in the experimental group was less than in the control group on day 1 (13.40 &lt; 27.60), day 3(12.65 &lt;, 28.35) and day 5 (11.78 &lt;29.23) post-operatively (Figure 2).&nbsp;</div>\r\n<div>\r\n	Analysis of PRI Within group on Day 1, 3 and 5</div>\r\n<div>\r\n	The PRI was not normally distributed for analysis pain score within groups using Friedman test, the result showed there was significantly different pain score (PRI) on day 1, day 3 and day 5 at 0.05 level (P =0.00). Therefore, with this result and compared mean rank of three days (day1&gt;day3&gt;day5) (2.44&gt;2.25&gt;1.31), it can be concluded that pain reduced over time (Table 3).&nbsp;</div>\r\n<div>\r\n	Analysis of VAS between Two Groups on Day 1, 3 and 5</div>\r\n<div>\r\n	The Visual Analogue Scale (VAS) was not normally distributed for pain analysis score between the two groups. When Mann-Whitney test &nbsp;was used, the results showed there was a significantly different pain score between the experimental and control group at 0.05 level on day 1 (P =0.00) and day 5 (P =0.00) &nbsp;post-operatively using VAS. It can be concluded that pain in the experimental group was less than in the control group on day 1 (13.23&lt; 27.78) and day 5 (14.20&lt; 26.80) post-operatively. There was no significant difference between the two groups on day 3 (P =0.09) but the mean rank score of experimental group was less than the control group (17.52&lt;, 23.48) (Figure 3).&nbsp;</div>\r\n<div>\r\n	Analysis of VAS Within group on Day 1, 3 and 5</div>\r\n<div>\r\n	The VAS was not normally distributed for analysis pain score within groups using Friedman test, the result showed there was significantly different pain score (VAS) on day 1, day 3 and day 5 at the 0.05 level (P =0.00). Therefore, with this result and compared mean rank for three days (day1&gt;day3&gt;day5) (2.35 &gt; 2.17&gt; 1.48), it can be concluded that pain reduced over time (Table 3).</div>\r\n<div>\r\n	Analysis of PPI between Two Groups on Day 1, 3 and 5</div>\r\n<div>\r\n	The Present Pain Intensity (PPI) was not normally distributed for analysis pain score between the two groups using Mann &ndash; Whitney test, the results showed that there was significant difference in pain score between experimental and control group at 0.05 level on day 1 (P = 0.029) and day 5 (P = 0.001) post-operatively, using PPI. Therefore, with the above results and compared mean rank between two groups, it could be concluded that pain in the experimental group was less than control group on day 1 (16.83 &lt; 24.18) and day 5 (15.00&lt;26.00) post-operatively. There was no significant difference between the two groups on day 3 (P = 0.557) but the mean rank score of the experimental group was less than the control group (19.52&lt;, 21.48) (Figure 4).&nbsp;</div>\r\n<div>\r\n	Analysis of PPI Within group on Day 1, 3 and 5</div>\r\n<div>\r\n	The PPI was not normally distributed for analysis of pain score within groups using Friedman test. The results showed that there was a significant difference in pain score (PPI) on day 1, day 3 and day 5 at the 0.05 level (P =0.000). It can be concluded that with the above result and the compared mean rank of three days (day1&gt;day3&gt;day5) (2.26&gt;2.21&gt;1.53), that pain reduced over time (Table 3).</div>\r\n<div>\r\n	Comparison of Paracetamol, Celecoxib and Tramadol Usage between Two Groups</div>\r\n<div>\r\n	The analgesic was not normally distributed using Mann&ndash;Whitney test, the result showed there was no significant difference on using Paracetamol, Celecoxib or Tramadol between experimental and control group for five days post-operative at the 0.05 level. Despite this view, compared median showed experimental group used less analgesic than control group post-operatively (paracetamol (16000.00 &lt; 17000.00), Celecoxib (600.00 &lt; 1600.00) and Tramadol (125.00 &lt; 225.00) (Table 4).</div>\r\n<div>\r\n	Additional pain description results</div>\r\n<div>\r\n	The pain feeling was one of the foundations of this study and the interesting result showed 97.5% to 82.5% had the pain feeling of heavy and tenderness. Hot burning and aching were the two most descriptive with 40% and 30%. None of the patients complained of shooting and punishing-cruel pain.&nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The results of the study showed that music therapy reduced post-operative pain after TKR. It was found that the pain score in the experimental group was statistically significantly less than control group on day 1 and day 5 by using PRI, VAS and PPI. The result was similar with other research findings that explained post-operative music therapy had positive effect on post-operative pain (Masuda et al. 2005; Laurion &amp; Fetzer 2003; Sendelbach et al. 2006; Voss et al. 2004).</div>\r\n<div>\r\n	Pain score on day 3 post-operatively using the VAS and PPI was not significantly different but when these were compared, the mean rank showed that the experimental group suffered less pain than the control group. The possible reason for worsening pain on day 3 could be due to reduced duration of listening, reduced concentration of listening and increased pain due to ambulation. The same result was found in a study by Vaajoki et al. (2012). They found significant difference in the patients&rsquo; pain intensity and pain distress on bed rest, during deep breathing and on shifting position on the second post-operative day but there was no significant difference on pain intensity and distress on day 3.&nbsp;</div>\r\n<div>\r\n	The reason for no difference in pain score between the two groups on day 3 could possibly be due to patient&rsquo;s concentration on the music. The researcher could not be sure that the patient concentrated on the music or was thinking of something else. According to Simcock et al. (2008), they found that music therapy had positive effect on post-operative pain but sometimes patient listened to music for only 60% of the time. This could reduce the effect of music therapy on pain (Simcock et al. 2008). &nbsp;The study totally depended on the patients&rsquo; intent to listen to the music and the nurses and researcher checked on them on and off but there was no accurate calculation that the patients listened for four hours daily. On post-operative day 3, patients usually started walking using the walking frame and this could affect the increasing pain rather than when patient was on rest position.</div>\r\n<div>\r\n	Another interesting finding of this study was that the pain score within groups was reduced over time by using PRI, VAS, and PPI (Day 1 &gt;day 3 &gt;day 5). The commonly used drugs on post TKR were Paracetamol, COX 2 inhibitors (Celecoxib) and opioid drugs (Tramadol) for five days post- operative. The results showed there was no statistical difference between experimental and control group who required analgesic but when median rate was compared it was clear that the experimental group used less analgesic compared to the control group. Many studies have explained that music therapy could not reduce usage of analgesic post-operatively (Sendelbach et al. 2006; Voss et al. 2004). In spite of this, some research explained that music therapy could reduce the usage of post-operative analgesic (McCaffrey &amp; Locsin 2006; Ebneshahidi &amp; Mohseni 2008; Tse et al. 2005; Pellino et al. 2005).</div>\r\n<div>\r\n	Patients were distributed equally between the two groups according to demographic data except for race and religion. Therefore, this study had unequal race distribution where experimental group had 15 Malays, 2 Chinese and 3 Indians, and the control group had 15 Chinese, 5 Malays and no Indians. The data was a randomized distribution between the two groups for improving accuracy and to reduce bias. If the race had been normally distributed between the two groups the result of pain score may have changed.</div>\r\n<div>\r\n	On the first day, the patients were not very interested in the music but on day 5 they changed and explained that the private room was rather boring without music. Majority of patients explained that music therapy was effective in reducing their pain and were feeling comfortable and relaxed. In one study, patients reported they were not sure that music could have an effect on reducing their pain but they believed that music therapy could divert their mind from the surgery (Simcock et al. 2008). In another study they had positive patient report in the treatment group, they mentioned that the interventions were helpful for pain (96%), and majority (64%) mentioned that their pain reduced to a moderate amount or a lot and increased their feeling of being in control of pain (62%) (Good et al. 2005).</div>\r\n<div>\r\n	Limitation of the Study</div>\r\n<div>\r\n	This study was a Quasi-experimental design that took every patients who were admitted and underwent the procedure of TKR in UKMMC at that particular time. This research population may not represent the whole population of Malaysia. Secondly, the ethnic group was not normally distributed between experimental and control group because this study used randomized technique for dividing the patients into two groups. Thirdly, communication was a &nbsp;problem, Malaysia has different races and languages, therefore some patients could not speak English or Malay, so they were omitted from this study. Information bias could also have happened; some patients may not have related their exact experience of pain.</div>\r\n<div>\r\n	Nursing Implication</div>\r\n<div>\r\n	This study recommended listening to the music as one of the non-pharmacological intervention that could reduce pain and could be more effective when used with analgesic drugs. Music therapy is cheap, available, safe and without side effects and are interventions that the nurses could use directly for reducing pain. Nurses play an important role in providing health care to patients. They are the ones in the frontline and receive the initial complains about pain from patients. It is important that nurses know that it is not only analgesics but music therapy too could help alleviate pain among patient. It is hoped that music therapy will be adopted as one of the non-pharmacological interventions for pain among patients in the wards eventually.</div>\r\n<div>\r\n	Recommendation</div>\r\n<div>\r\n	Non pharmacologic intervention is cheap, safe and with no side effects. Many studies have suggested that combination of non-pharmacological intervention with pharmacological intervention can reduce pain better instead of by only using drugs. Therefore, this study suggests that usage of different types of non-pharmacological intervention such as music therapy for reducing pain, can increase patient satisfaction and comfort. Music therapy is not common as non-pharmacological intervention in Malaysia and it is expected that in future, elaborate research on the effect of music therapy on post-operative pain in different areas could be carried out. Many studies support that music therapy can reduce anxiety and stress, so it will be interesting if soothing and relaxing music can be used in the ward environment and these may have a positive effect on patients and nurses.&nbsp;</div>\r\n', 1),
(1355, 1346, '<div>\r\n	Case Report&nbsp;</div>\r\n<div>\r\n	Case 1</div>\r\n<div>\r\n	A 34-year-old man motorcyclist was hit by a lorry rear bumper over his anterior chest wall. He presented to Emergency Department (ED) of a university hospital two hours after the accident and complained of moderate chest pain with verbal numerical rating score (VNRS) of seven. Pain worsened on inspiration and it radiated to the back. His blood pressure (BP) was 190/114 mmHg, heart rate (HR) was 83 per minute, afebrile, oxygen saturation under room air (sPO2) was 98% and respiratory rate (RR) of 19 per minute. Other systemic examinations were unremarkable. Extended Focal Abdominal Sonography for Trauma (eFAST) found no abnormality. Bedside echocardiography showed no abnormality. Chest X-ray was normal. Initial electrocardiography (ECG) showed ST segment elevation of lead V2 and V3 (J point = 3mm elevation) and T inversion over lead I and V6 (Figure 1). The initial creatine kinase (CK) was minimally raised (169 U/L), but troponin T was normal. He was administered intravenous tramadol and the chest pain resolved (VNRS zero). Patient was admitted to ED short-stay ward (EDSSW) for 24 hours observation. The subsequent serial of ECG&rsquo;s did showed resolution of ST-segment elevation to baseline at lead V2 &amp; V3 with persistent T inversion at lead I &amp; V6. Repeated CK after 6 hours showed no further increment and the troponin T still remained undetectable. Patient was discharged after 24 hours of observation. The ECG on discharge showed normalised ST segment and T wave in all leads (Figure 1). CK level repeated three days later was 285 U/L. The troponin T and ECG remained normal.</div>\r\n<div>\r\n	Case 2</div>\r\n<div>\r\n	A 21-year-old male car driver was brought in to ED after a head on collision with another car. He did not fasten his seatbelt. His anterior chest hit the steering wheel. He presented to ED of with the complaint of right sided headache &amp; central chest pain with VNRS of eight. His BP was 127/67 mmHg, HR was 88/min, RR was 18/min and sPO2 was 97% under room air. His Glasgow Coma Scale score was 14 out of 15. There were multiple abrasion wounds over his face and tenderness over the right iliac fossa. Other systemic examinations were unremarkable. &nbsp;Initial ECG showed ST segment elevation on lead V2 (J point = 3mm elevation) and V3 (J point = 1.5mm elevation) (Figure 2). &nbsp;EFAST examination showed no abnormality. Computer tomography of brain and cervical spine were performed and reported as normal. His initial CK level was raised (265 U/L) but troponin T level was undetectable (&lt;0.013ug/L). Other blood investigations were normal. He was given intramuscular anti-tetanus toxoid and intravenous analgesia. The chest pain was resolved (VNRS zero). He was then admitted to EDSSW for cerebral concussion care and cardiac concussion monitoring. The serial of ECG had shown resolution of ST segment V2 and V3 to baseline (Figure 2). A repeat CK and troponin T after 14 hours post-accident showed mild elevation of CK level (306 U/L) and troponin T remained undetectable. He was discharged well after 24 hours of observation in EDSSW. A follow-up telephone call to patient was made three days later showed uneventful recovery.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	There are cases which do not fit in the current cardiac concussion definitions domain which are illustrated by the above two cases. These patients have regional ECG changes. They do not fulfil the diagnosis of cardiac concussion (commotiocordis) whereby the ECG changes are global. Therefore, the diagnosis of &ldquo;focal&rdquo; cardiac concussion should be considered. The current practice in diagnose and manage &ldquo;focal&rdquo; cardiac concussions are mainly based on acute coronary syndrome (ACS) modality. In these cases, the history and significant changes in the anterior ECG leads may indicate that they had cardiac concussion localized to the anterior wall of the heart. Linton et al. described the elevation of the ST segment indicates direct contact of a specific area of myocardium (Bishop et al. 1956). However, &ldquo;focal&rdquo; cardiac concussion differs from ACS in term of pathophysiology. &ldquo;Focal&rdquo; cardiac concussion involved electrophysiological component compare to ACS, which involved both myocardium perfusion and electrophysiological changes Therefore, the definition of &lsquo;focal&rsquo; cardiac concussion should take into account both electrophysiological and microcellular pathophysiological changes.&nbsp;</div>\r\n<div>\r\n	Existing biomarker of cardiac damage is based on injury to the cardiac muscle. They may not necessarily reflect dysfunction of the electrophysiological component of the myocardium. Nonetheless, immune-histochemical changes may be present (Dettmeyer 2011). In a canine experimental study, patchy focal loss of the myocardial myoglobin, CK-MM and CK-BB were identified with these substances scattered deposited of between myocardial fibres (Dettmeyer 2011). The CK levels were elevated with normal troponin T level. The diagnosis of focal cardiac concussion maybe more appropriate in cases of regional ST segment elevation in ECG with normal troponin T level. A new diagnostic modality and approach should be developed. Further experimentation is recommended to develop a more accurate definition of cardiac concussion. In terms of patient&rsquo;s disposition, there is a dilemma as the prognosis is uncertain. Therefore, managing this group of patients is rather challenging. They were both successfully managed in EDSSW. Thus, it reduces in&ndash;hospital admission and can be cost effective.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	We propose the diagnosis of focal cardiac concussion for these two cases. We also highlighted the ability of ED to manage these patients in EDSSW.</div>\r\n<div>\r\n	Acknowledgement</div>\r\n<div>\r\n	The authors thank the doctors, assistance medical officer, nurses, technical and administrative support staff in the Emergency Department, Universiti Kebangsaan Malaysia for their assistance in this study.</div>\r\n', 1),
(1358, 1349, '<div>\r\n	QUESTION</div>\r\n<div>\r\n	A 4-year-old child was brought to the Emergency Department with generalized&nbsp;</div>\r\n<div>\r\n	tonic-clonic seizures. Right hemiparesis and unequal pupils were noted during examination. Computed Tomography of brain revealed acute subdural hemorrhage for which an emergency craniotomy and evacuation of clot was performed. Intraoperative finding of abnormal vessels prompted a need to revisit his past surgical history. Three years ago, he was diagnosed with hydrocephalus secondary to cerebral aqueduct stenosis, which eventually led to the insertion of a ventriculo-peritoneal shunt. Since then, he has suffered from global developmental delay and infrequent episodes of epilepsy. One month prior to this recent presentation, he was treated for varicella zoster infection and discharged well, five days later. The initial Magnetic Resonance Imaging of brain taken three years earlier is also shown in Figure 1.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	What should be the more appropriate cause of hydrocephalus in this patient?</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Is there a correlation between varicella zoster infection and the current presentation of acute subdural hemorrhage?</div>\r\n<div>\r\n	Answer and Discussion:&nbsp;</div>\r\n<div>\r\n	Vein of Galen named after the Roman Physician Galen of Pergamon is formed by the union of basal veins of Rosenthal and the internal cerebral veins (de Tribolet 2009). Vein of galen malformation (VGAM) accounts for 30% of the cerebral vascular malformationin the pediatric population, and is caused by teratogenic insult occurring between the 6th to 11th week of intrauterine life. The failure of median prosencephalic vein to obliterate leads to aneurysmal dilatation of vein of galen and arterio-venous shunting of blood (Gailloud et al. 2005). The diagnosis is often delayed due to non-specific clinical presentation and low index of suspicion. Cardiac failure is the most common clinical presentation followed by hydrocephalus and intracranial hemorrhage (Suh et al. 2001). Third trimester ultrasonographic evidence of dilated median prosencephalic vein or cardiomegaly is suggestive of VGAM. Digital subtraction angiography remains the gold standard radiological investigation. It is used to confirm diagnosis and differentiate aneurysmal dilatation from arterio-venous malformation. Transarterial occlusion of feeder vessels have dramatically improved prognosis while reducing surgery related mortality. Seizures and parenchymal damage is a sign of poor functional outcome. Once left untreated, the mortality rate is almost 100% (Gupta et al. 2004). To the best of our knowledge, there is no documented correlation between VGAM and varicella zoster infection. However, varicella zoster-induced vasculitis presenting as intracranial haemorrhage and stroke have been reported in adults. The exact pathophysiology remains unclear but few suggest direct viral invasion into the endothelium as a plausible cause for inflammation, stenosis and occlusion (Jain et al. 2003).&nbsp;</div>\r\n', 1),
(1364, 1355, NULL, NULL),
(1716, 1707, NULL, NULL),
(6045, 6036, '<div>\r\n	Statin is a drug used to lower cholesterol level. It was first approved by the US FDA in 1987 and has since become a very popular drug to prevent cardiovascular diseases related to high cholesterol levels. In 1999, Mundy and his co-researchers discovered another interesting property of statin. After screening thousands of compounds, statin was the only compound found to promote bone morphogenetic protein (BMP) gene expressions and enhanced new bone formation in vitro and in animal model. This has caused excitement among the scientific community since very few agents were approved to treat osteoporosis, even though the number of osteoporosis cases was on the rise with the ageing society. This was rather a special discovery since there are not many drugs which have dual therapeutic effects, as seen in the case of statins i.e. lowering cholesterol and treating osteoporosis. Both these indications may seem to be unrelated, but further studies have revealed that adipose tissues (fat cells) and osteoblasts (bone-forming cells) originated from the same precursor stem cell i.e. the mesenchymal stem cell. It was noted that reduction in the formation of adipose cells from the mesenchymal stem cell resulted in the increase in the formation of osteoblast.</div>\r\n<div>\r\n	With regard to mechanism of action, statin works by inhibiting HMG-CoA reductase, the rate-limiting enzyme in the cholesterol biosynthetic pathway to reduce cholesterol synthesis. The inhibition of this enzyme may also increase the production of BMP, thus increasing bone formation and preventing bone loss and osteoporosis.</div>\r\n<div>\r\n	Several animal studies reported that statins protected bone against osteoporosis and promoted fracture healing of osteoporotic bone. Human studies were conducted on patients taking statins by measuring their bone mineral densities and assessing their risks of fractures. There were mixed results on these human studies. According to a systematic review by Salari and Abdollahi (2011), more longitudinal studies in different ethnicities with large sample size are required before any conclusion could be drawn on statin use as anti-osteoporotic agent. The question arises on why the data from human studies on statin&rsquo;s anti-osteoporotic effects were not convincing. The answer may lie in the oral doses of statins used. In most subjects, statins were taken according to the doses recommended to lower cholesterol.</div>\r\n<div>\r\n	Statins are degraded by the liver, resulting in low bioavailability to reach the bone environment. Abdul-Majeed et al. (2012) commented that the anti-osteoporotic effects could only be seen with very high oral doses of statins as they are extensively degraded in the liver. This may not be practical as high doses of statin were associated with risks of liver failure and rhabdomyolysis. Therefore, a new approach of delivering statins to the bone is required. Recently, Ibrahim et al. (2014) reported that statin that was delivered directly to bone via targeted and controlled delivery system, promoted fracture healing in osteoporotic bone of post-menopausal model. By using this delivery system,&nbsp;89a low dose of statin was combined with a polymer and injected directly to the fracture site. This has opened up a new area of research on statin as an anti-osteoporotic agent. Further studies on statins are advised to determine its potential as anti-osteoporotic agent.</div>\r\n', 1),
(6035, 6026, NULL, NULL),
(6042, 6033, NULL, NULL),
(6052, 6043, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Crab is an example of shellfish which contributes an important source of proteins for humans. Scylla serrata, also known as mud crab is highly demanded as a protein food source by various communities in China, Vietnam, Singapore, Taiwan, Hong Kong and Malaysia (Keenan 1999). However, in Malaysia, the mud crab industry is still not making headway and is only carried out on a small scale by local fishermen. From the annual production, over 1.3 million tonnes were contributed by the local fishery industry (FAO 2010).</div>\r\n<div>\r\n	However, crab can also cause seafood allergy (Abramovitch et al. 2013; Hsu &amp; Li 2012; Ma et al. 2012; kinaseHuang et al. 2010). In Malaysia, the prevalence of shellfish allergy, one of which includes crab, was reported to be 44% among patients with allergic rhinitis and asthma (Shahnaz et al. 2001). Most crab species, provoking allergic reaction including the skin, gastrointestinal, respiratory and cardiovascular systems (Lopata &amp; Lehrer 2009; Liang et al. 2008; Fontan et al. 2005). Exposure to crab allergens may occur through ingestion of crab diet and inhalation or skin contact while processing, cooking or working (Butt &amp; Macdougall 2008).</div>\r\n<div>\r\n	To date, several clinically relevant allergens of crab have been identified (Abdul Rahman et al. 2011; Liang et al. 2008; Lehrer et al. 2003).</div>\r\n<div>\r\n	<div>\r\n		Tropomyosin, a 34-38 kDa protein has been demonstrated to be the major allergen for crabs (Rosmilah et al. 2012; Shriver &amp; Yang 2011; Abdul Rahman et al. 2011; Liang et al. 2008; Lehrer et al. 2003) and is believed to be a pan-allergen of invertebrates including arthropods and mollusks (Suma et al. 2007; Lehrer et al. 2003; Ayuso et al. 2002; Chu et al. 2000). Apart from tropomyosin, arginine kinase was also identified in mud crab (Yu et al. 2013; Liu et al. 2012; Shen et al. 2011). Tropomyosins are a family of closely related proteins present in muscle and non-muscle cells and, in association with the troponin complex, they play a central role in the actin-linked calcium regulatory system of muscle contraction in all invertebrates and vertebrates (Reese et al. 1999). Arginine kinase is an enzyme that reversibly catalyzes the transfer of phosphate between ATP and various phosphogens in all invertebrates (Abdel Rahman et al. 2011).</div>\r\n	<div>\r\n		Several additional new allergens of crab were identified as sarcoplasmic calcium-binding protein (20 kDa), troponin (23 kDa), &alpha;-actine (42 kDa), smooth endoplasmic reticulum Ca2+ATPase (113 kDa) (Abdel Rahman et al. 2011) and myosin light chains (Abramovitch et al. 2013).</div>\r\n	<div>\r\n		Since, there is scarcity of information on characterization of allergens of this species of crab, the present study was conducted to compare the IgE-binding proteins of raw and cooked extracts of the mud crab (Scylla serrata) among the local population with crab allergy.</div>\r\n	<div>\r\n		MATERIALS AND METHODS PREPARATION OF CRAB EXTRACT</div>\r\n	<div>\r\n		Live mud crab was purchased from an aquaculture centre in Port Klang, Selangor. Raw and cooked mud crab extracts were prepared according to procedures described by Rosmilah et al. (2012). Briefly, the crab meat was washed with purified water and then homogenized in phosphate buffered saline (PBS) with pH 7.2 for 3 mins. The homogenates were then extracted overnight at 4˚C under a constant mixing using a shaker, followed by centrifugation at 4˚C at 4500 rpm for 30 mins and at 14,000 rpm at 4˚C for another 30 mins. The clear supernatant was filtered using syringe filter 0.20 &mu;m. Finally, the supernatant was transferred to 1.5 ml eppendorf tubes before lyophilizing using freeze dryer and then stored at -20˚C until use. For the cooked extract, the homogenates were boiled for 10 mins before extraction.</div>\r\n	<div>\r\n		PATIENTS&rsquo; SERA</div>\r\n	<div>\r\n		Sera of 21 patients with history of mud crab allergy and positive skin prick test (SPT) to mud crab extract were used in this study. Skin Prick Test (SPT) was performed by medical officers at Allergy Clinic of Kuala Lumpur General Hospital. Serum from a non-allergic subject was used as a negative control. Ethical approval was obtained from the Medical Research and Ethics Committee (MREC), Ministry of Health, Malaysia.</div>\r\n	<div>\r\n		<div>\r\n			SODIUM DODECYL SULFATE-POLYACRYLAMIDE GEL ELECTROPHORESIS (SDS-PAGE)</div>\r\n		<div>\r\n			SDS-PAGE was performed according to the procedure of Rosmilah et al. (2012). Mud crab extracts were heated at 97˚C for 4 mins in a denaturing Laemmli buffer before resolving into 12.5% separating gel with a 5% stacking gel by using a Mini Protean 3 apparatus (BioRad, USA) at 120mA for 45 mins. Precision Plus Protein Dual Color Standard (BioRad, USA) was used as a marker. The gel was then stained with Coomassie brilliant blue (CBB) R-250. The molecular weight of each protein was estimated by comparing the protein&rsquo;s gel position to the protein markers (Biorad, USA) in the same gel using an imaging densitometer (BioRad, USA).</div>\r\n		<div>\r\n			IMMUNOBLOTTING</div>\r\n		<div>\r\n			Immunoblotting technique was carried out as described by Rosmilah et al. (2012) using sera from 21 patients as described above. Protein samples subjected to SDS-PAGE electrophoresis were transferred onto nitrocellulose membrane using a Mini Transblot System (BioRad, USA) at 100 V for 70 mins. Each membrane was stained with Ponceau S (Sigma, USA) to verify transfer of the separated proteins. Then, the membrane was cut into 3 mm width followed by washing them with TTBS (containing 0.05% Tween 20). Non-specific protein sites were blocked with 5% of non-fat milk in TBS acting as a blocking buffer. Then, the strips were incubated in the test sera at 4˚C overnight with a shaker. The IgE-binding on the strips were detected by incubation with biotinylated goat antihuman IgE (Kirkergaard and Perry Laboratories, UK) and streptavidin-conjugated alkaline phosphatase (BioRad, USA) that encourage labelling the antibody for 30 mins at room temperature. Finally, an Alkaline Phosphatase Conjugate Substrate Kit (BioRad, USA) was used to detect the IgE binding proteins by incubating the strips with the substrate for 10 mins at room temperature. Each plate or set of strips contained a blank (no serum) and a negative control (normal serum).</div>\r\n		<div>\r\n			RESULTSSDS-PAGE OF MUD CRAB EXTRACTS</div>\r\n		<div>\r\n			SDS-PAGE of raw mud crab extract revealed 20 protein bands with molecular masses ranging from 12 to 250 kDa. Both raw and cooked extracts afforded a prominent heat-stable protein band of 36 kDa. Protein bands between 40 to 250 kDa were absent in cooked extract compared to raw extract due to heat denaturation. Hence, they were identified as heat-sensitive proteins.</div>\r\n		<div>\r\n			REACTIVITY OF IGE IN PATIENTS&rsquo; SERA WITH MUD CRAB EXTRACTS</div>\r\n		<div>\r\n			Immunoblotting experiment using 21 sera from mud crab allergic patients demonstrated several IgE-binding proteins at various molecular masses between 23 to 250 kDa (Figure 1). Generally, more IgE-binding was detected in the immunoblotting of raw extract compared to cooked extract. A heat-resistant 36 kDa protein was identified as the major allergen of mud crab with frequencies of 95% in both extracts. In addition, a heat-sensitive protein of 41 kDa has also been recognized as a major allergen in raw extract. Other heat-sensitive proteins of 25, 28, 65, 75 and 95 kDa were also recognized as potential minor allergens (frequencies of more than 25%) only in raw extract. IgE-binding profiles of all sera tested were summarized in Table 1. No IgE-binding protein was detected in the negative control and blank.</div>\r\n		<div>\r\n			DISCUSSION</div>\r\n		<div>\r\n			Previous studies reported the prevalence of shellfish allergy to be common among local population in Malaysia with allergic rhinitis and asthma (Shahnaz et al. 2001).</div>\r\n		<div>\r\n			As observed in SDS-PAGE gels, mud crab extracts contain various heat-stabile and heat-labile proteins ranging from 12 to 250 kDa. The raw crab extract revealed 20 protein bands while the cooked extract yielded smaller number of bands compared to raw extract due to denaturation of several protein bands in the range of 40 to 250 kDa. The disappearance of bands in cooked extract may be due to loss of some protein structures. Previous studies showed heat denaturation of globular proteins may disrupt their tertiary structure and lead to random-coiled aggregation and insolubility (Peng &amp; Gygi 2001). Therefore, alteration in protein structure by food processing may lead to epitope destruction, modification, masking or unmasking thereby decreasing, increasing or having no effect on allergenicity (Sathe et al. 2005). In the present study, we detected several heat-resistant and heat sensitive proteins ranging from 23 to 250 kDa as IgE-reactive proteins.</div>\r\n		<div>\r\n			Many food allergens are resistant to denaturation, which allows them to retain the ability to elicit allergic reactions under conditions such as heating and enzymatic degradation. Thus, proteins that are resistant to high heat or digestive conditions may enter the gastrointestinal tract with unaltered epitopes and cause allergic reactions (Shriver &amp; Yang 2011). This is observed in tropomyosin, a 34 to 36 kDa heat-resistant major allergen in numerous invertebrates, including crabs (Rosmilah et al. 2012; Shriver &amp; Yang 2011; Abdul Rahman et al. 2011; Liang et al. 2008; Lehrer et al. 2003). Interestingly, in this study, we found that 95% of patients exhibited IgE-binding proteins at 36 kDa in both extracts. Thus, it is possible that our 36 kDa may be due tropomyosin. Several sera demonstrated the enhancement of IgE-binding intensity to the tropomyosin protein (36 kDa) in immunoblotting of cooked extract indicating increased allergenicity. This finding was similar to other reports which suggested that tropomyosin was more allergenic in boiled forms (Rosmilah et al. 2012; Yadzir et al. 2010; Liu et al. 2010a; Besler et al. 2001; Nagpal et al. 1989), probably because boiled tropomyosin may have undergone protein-protein interactions such as aggregation during the thermal treatment to cause enhanced IgE binding. Therefore, the boiled tropomyosin seems to be more effective in diagnosing of seafood allergy (Liu et al. 2010b).</div>\r\n		<div>\r\n			Aside from tropomyosin, arginine kinase with molecular weight of 40 kDa has also been identified as a significant allergen in crabs (Shen et al. 2011; Abdel Rahman et al. 2011; Yu et al. 2003; Hendrickx et al. 1998). Notably, enzymes including arginine kinase are sensitive to high temperature and will lead to denaturation (Hendrickx et al. 1998). In the present study, we found that 57% of our subjects had binding to the 41 kDa band, which may be arginine kinase. However, the mechanism of allergic sensitization to arginine kinase is unknown (Yu et al. 2003). Although, it was reported that purified arginine kinase may bind IgE antibodies in in vitro tests and induce allergic reactions in sensitized patients, the clinical relevance and the functional relationship between arginine kinase activity and allergenicity may require further investigation (Yu et al. 2003). Moreover, other heat-sensitive proteins at 25, 28, 65, 75 and 95 kDa were detected as potential minor allergens in raw extract. In the present study, minor allergens were also observed at various molecular masses. However, more detailed identification and characterization of these proteins need to be performed.</div>\r\n		<div>\r\n			CONCLUSION</div>\r\n		<div>\r\n			The results of the present study showed that mud crab has two major allergens, a heat-resistant protein of 36 kDa and a heat-sensitive protein of 41 kDa. Further studies using allergenomic approaches will be carried out to identify the biochemical properties of these major allergens.</div>\r\n		<div>\r\n			ACKNOWLEDGEMENT</div>\r\n		<div>\r\n			The authors wish to thank the Director General of the Ministry of Health of Malaysia for the permission to publish this paper. This study was supported by the research grant (FRGS 2010-0065-102-02).</div>\r\n	</div>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(6053, 6044, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Stroke represents the third most common cause of death in developed countries (Mackay &amp; Mensah 2004). &nbsp;It is frequently associated with higher risk for a wide range of physical and neuropsychological consequences (Bourgeois et al. 2004). One of the post-stroke psychiatric comorbidity is mood disorders, especially depression (Lai et al. 2002). Post- stroke depression (PSD) has been reported occurred in 11% to 75% of all stroke patients (House et al. 1991). The major depression was the one most affected post stroke survivors within first few months after stroke. It is about 10% to 27 % compare to minor depression 15% to 40% (House et al. 1991). In addition, a study conducted by Glamcevski and Tan (2000) at University Malaya Medical Centre in Malaysia which involved 80 post-stroke patients. A total of 53 (66%) had depression, and of these, 51% were mild, 11% were moderate and 4% were severe. Research on depression following stroke has gained momentum but it is still uncertain with relation to depression after stroke with clinical characteristics. Therefore, the present study was carried out to observe the relationship between clinical characteristics and the levels of depression among post-stroke survivors.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Subjects&nbsp;</div>\r\n<div>\r\n	This study was conducted among consecutive stroke survivors who visited a rehabilitation clinic at one of the teaching hospital in Kuala Lumpur, Malaysia. The participants were recruited from October 2009 till January 2010. Only patients with first-time stroke whose CT scan showed lesions compatible with their neurological symptoms, were included. Prior to the data collection, the researcher performed a screening of the patients using the Mini Mental State Examination (MMSE) to assess their cognitive ability. Patients who were severely impaired of cognitive function (MMSE&lt;24), severe aphasia, language problems (unable to understand the Malay, English or others) even after being assisted by researcher or the patient&rsquo;s proxy, children with stroke, stroke patients with other neurological problems, and those with less than one month post-stroke duration, were excluded from this study.</div>\r\n<div>\r\n	Instruments&nbsp;</div>\r\n<div>\r\n	All patients and their proxy were thoroughly informed about the study and written consent was obtained. The study was approved by the Medical Research Ethics Committee of Universiti Putra Malaysia. Those met study criteria were interviewed to assess their level of depression by using Beck&rsquo;s Depression Inventory (BDI). The BDI has been used for 35 years to identify and assess depressive symptoms, and it has been reported by Beck et al. (1961) to be highly reliable regardless of the population. It has a high coefficient alpha (.80), its construct validity has been established, and it has an ability to differentiate the depressed from non-depressed patients. In Malaysia, the BDI Malay version was tested and the internal consistency (Cronbach&rsquo;s alpha) ranged from = .71 to .91 (Mukhtar &amp; Oei 2007). Each item was evaluated on a severity scale ranging from 0-3, with a total score ranging from 0-63. The data on patients&rsquo; demographic and clinical characteristics were retrieved from the patient&rsquo;s medical record.&nbsp;</div>\r\n<div>\r\n	Data Analysis</div>\r\n<div>\r\n	All the data collected for this study was analyzed by using the Statistical Package for Social Sciences (SPSS) version 16.0 for Windows&reg;. Descriptive statistics was used to determine the socio-demographic characteristics (age and gender) and clinical characteristics (stroke types, post-stroke duration ,stroke severity, and levels of depression). A two-tailed probability value of p &lt; 0.05 was used to identify the criterion for statistical significance.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Clinical characteristics</div>\r\n<div>\r\n	Levels of depression&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The relationship between clinical characteristics and the levels of depression&nbsp;</div>\r\n<div>\r\n	A Chi-square test for independence was performed to determine the relationship between clinical characteristics (post stroke duration, stroke type and stroke severity) and the levels of depression. The test indicated that there was significant association between stroke severity with levels of depression, &chi;2&sup2; (2, n=195) = 28.724, p &lt; 0.001, phi = 0.384. Stroke survivors with mild stroke had less depression (44.0 %) compared to those with moderate stroke (81.7 %) and severe stroke (87.5%). &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The BDI scores in this study indicated 59.5% of post stroke survivors had depression. The severe depression represented 16.4% of the total participants. According to Glamcevski and Tan (2000) &nbsp;depression is common among post-stroke survivors. This is because of stroke attributed to the deterioration of physical functioning among post-stroke survivors (Bendz 2003). The finding of study is also similar to the study reported by Glamcevski and Tan (2000) who also found that 66.0 % of post-stroke survivors had depression. Furthermore, recent stroke literature also reported depression among post-stroke survivors was 30%-35% (Williams 2005). Therefore, it is not too surprising to find a 59.5% depression rate in this subject population. &nbsp;</div>\r\n<div>\r\n	In this study, there was a significant relationship between stroke severities with depression. Whereby, the post- stroke survivors with severe stroke had severe depression compared to the moderate and mild. This was because those post-stroke survivors with severe stroke had more physical inability which resulted in inability to function normally. They were more independent to others for their daily activities which resulted in affection of their mood changes especially psychological. Thus, this increased their depression level. Therefore, beside rehabilitation therapy, treatment after stroke should included for assessment of depression and treatment, accordingly. The family members should also be counselled and provided with necessary information on their post-stroke status.&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, this study found most the stroke survivors were depressed and the stroke severity had relationship with depression. Those with severe stroke were more depressed.&nbsp;</div>\r\n', 1),
(6057, 6048, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Since 1993, ultrasound has been an accepted tool for detection of life threatening intra-abdominal bleed among trauma patients in the ED (Sippel et al. 2011). With the inclusion of bedside ultrasound in the emergency master&rsquo;s curriculum (Bahner et al. 2013), the Emergency Physicians should be expected to perform ultrasound in the ED. Work by Fenkl et al. (1992) showed the ability of ultrasound to pick up sternal fractures. Bitschnau et al. (1997) showed good correlation between ultrasound and X-rays in detecting rib fractures.</div>\r\n<div>\r\n	Triage zone is the doorway entry to the ED. It aims to direct and ensure that patients are treated in the order of their clinical urgency. Triage also allows for the allocation of the patient to the most appropriate assessment and treatment area (Australian College for Emergency Medicine 2013). Ultrasonography is performed mostly in the critical and semi critical areas in the ED. However, the use of ultrasound is suboptimal in the triage area where we sort, direct and give initial treatment to patients in which critical initial diagnosis matters the most.&nbsp;</div>\r\n<div>\r\n	Triage area is unique and different from the rest of the ED where it plays a role as a front gateway. The triage environment has a limited space and time contact with patients. Previous studies assessed patients in the other areas of the ED or wards where space and time do not count as a critical issue in comparison to the triage area. The literature review conducted did not show any study specifically looking at ultrasound use as a triage tool in the triage environment of the ED. <span class="Apple-tab-span" style="white-space:pre"> </span>The aim of the present study was to assess the effectiveness of ultrasound to diagnose fractures in the triage area compared to plain radiographs. &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; Musculoskeletal related complaints are seen quite common among the non-critical patient who present to the ED. A study in Switzerland found that non-critical patients to the ED commonly had trauma related musculoskeletal complaints. Up to 32.7% of non-critical patients in the Swiss ED involved in their study had injuries related to the musculoskeletal system (Chmiel et al. &nbsp;2011).</div>\r\n<div>\r\n	Ultrasound usage by non-radiologists has been increasing over the years. Specialties such as surgery, orthopedics, and anesthesiology have started to use ultrasound in their practice for diagnoses and in guiding procedures (Blankstein 2011; Lindelius et al. 2008; Sites &amp; Antonakakis 2009). Ultrasound use has been explored in extreme environments like the international space station (Law &amp; Macbeth 2011). The ultrasound also shown promising results as an imaging modality using telemedicine &ndash; this has applications in rural areas, space medicine and disaster scenarios (Pian et al. 2013).&nbsp;</div>\r\n<div>\r\n	Ultrasound use has been explored in various body parts of the trauma patient. Investigators have explored ultrasound use to diagnose facial bone and nasal bone fractures in the trauma patient (Friedrich et al. 2003; Javadrashid et al. 2011). In fact, low dose pulsed ultrasound has been investigated as an agent to accelerate bone healing during fracture with mixed results (Busse et al. 2009). In the future, the Emergency Physician should be able to perform a comprehensive musculoskeletal assessment of the trauma patient, as advanced ultrasound skills enable the identification of ligament injuries, muscle tear (Lee &amp; Healy 2004) and fracture involving cartilaginous growth plates in children (Warkentine et al. 2014).</div>\r\n<div>\r\n	An evidence based review of bedside ultrasound stated that 50-95% of limb X-rays can be avoided in limb injured patients without missing fractures. This statement was based &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;on studies by authors such as (Heyworth 2003) which showed that only 15% of ankle injured patients had objective fractures in X-rays. Another study by Stiell et al. (1992), showed a fracture rate of only 4.3% mid foot and 9.3% malleolar in those who presented with ankle injuries. The use of ultrasound could be an adjunct to clinical decision rules in avoiding unnecessary X-rays in the patients such as those studied by Heyworth and Stiell above.</div>\r\n<div>\r\n	Clinician-performed bedside ultrasonography is emerging as a useful diagnostic tool for healthcare providers in resource-limited settings (Sippel et al. 2011). A recent study in Sweden &nbsp;showed that operators with minimal training were able to rule out ankle fractures using ultrasound (Hedelin et al. 2013).</div>\r\n<div>\r\n	In an earlier study, it was shown that Emergency Physicians with merely one hour of training, were able to achieve a sensitivity of 92.9% and specificity of 83.3% in diagnosing fractures of the humerus (Marshburn et al. 2004).</div>\r\n<div>\r\n	Another study showed that Pediatric Emergency Medicine Physicians with a one hour training were able to achieve a sensitivity of 73% (95% confidence interval [CI], 58%-84%), and specificity of 92% (95% CI, 86%-95%) in diagnosing long bone fractures of paediatric patients, defined as ages 1 &ndash; 25 years (Weinberg et al. 2010).&nbsp;</div>\r\n<div>\r\n	Fracture can be diagnosed by ultrasound as the bony cortex reflects sound waves, so the outer cortex and periosteum are well seen by ultrasound. A fracture shows up as a disruption of the cortical outline, hematoma at point of fracture, or avulsion fragments (Cross 2011).</div>\r\n<div>\r\n	The use of ultrasound in trauma is currently focused on Extended Focused Assessment of Sonography in Trauma (EFAST). If enough evidence accumulates, it will be justified to equip the triage area with an ultrasound machine to rule out fractures.</div>\r\n<div>\r\n	The present study aimed to assess the efficiency of ultrasound in diagnosing a fracture in the triage area as compared to X-rays. The gold standard in fracture diagnosis were the X-ray findings and the ultrasound diagnosis of the presence or absence of fracture which were compared to the X-ray findings. Increased use of ultrasound holds the promise of lower cost, faster time, and avoidance of the ionizing radiation present in X-rays.</div>\r\n<div>\r\n	Materials and Methods &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	This was a prospective cross sectional study. The location was within the triage area, Emergency Department, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). The study was carried out for a period of three months.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Inclusion criteria:</div>\r\n<div>\r\n	Patient showing any clinical signs and/or symptoms (swelling, tenderness, deformity, limited range of motion) of closed fracture.</div>\r\n<div>\r\n	Age above 18 years.</div>\r\n<div>\r\n	Exclusion criteria:</div>\r\n<div>\r\n	Open fracture.</div>\r\n<div>\r\n	Old fracture at site.</div>\r\n<div>\r\n	Deformed bone.</div>\r\n<div>\r\n	Data collection:</div>\r\n<div>\r\n	Non-critical patients presenting to the triage area of the emergency department with musculoskeletal complaints requiring imaging were selected. The study was explained to them with the assistance of the patient information sheet. If the patient was interested in participating in the study, they underwent the usual triage assessment, with the addition of ultrasound examination of the affected body area. The patient then underwent an X-ray and received the routine treatment as appropriate.&nbsp;</div>\r\n<div>\r\n	Data Analysis:</div>\r\n<div>\r\n	Data that was collected was analyzed using SPSS 12.0 for Windows [Release 12.0.0 (4 September 2003)].&nbsp;</div>\r\n<div>\r\n	Ethical aspects:</div>\r\n<div>\r\n	Ethical approval was obtained from the Ethics Committee (Ethics Committee/IRB ref no: UKM 1.5.3.5/244/FF-2014-311) of UKMMC. This study commenced once ethical approval was obtained. The established ethical principles were followed to ensure no harm would be caused &nbsp;to the participants. The researcher ensured adequate communication with the participants regarding the intention of the study.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Ultrasound methods:</div>\r\n<div>\r\n	Following was the standardized technique for assessing efficiency of fracture by ultrasound. Initially the procedure was explained to the patients and consent was taken. Patient details were recorded on data collection sheet. The linear ultrasound probe was covered with ultrasound gel and the probe was traced along the length of the suspected site of fracture. &nbsp;Positive or negative finding by ultrasound probe was recorded onto the data collection sheet. Scanned body part was cleaned of any leftover gel, and patient is sent for X-ray. Fracture was detected in the presence of these ultrasonographic features, which were disruption of bony cortical outline, presence of avulsion fracture fragments and hematoma at point of fracture. Figure 1 shows the ultrasonographic picture of fracture. One could observe the discontinuation of the cortical bone, and hematoma formation (anechoic-black colour) above the fracture. Figure 2 shows X-ray view.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Flow chart of data collection is depicted in Figure 3.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Sample size calculation (Kish, L. 1965):</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ss = Z2 * (p) * (1-p)</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;(S.E)2</div>\r\n<div>\r\n	Where:</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Z = Z value (e.g. 1.96 for 95% &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; confidence level)&nbsp;</div>\r\n<div>\r\n	p = percentage picking a choice, &nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; expressed as decimal (.5 used for&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; sample size needed)</div>\r\n<div>\r\n	S.E: standard error</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Z = 1.96</div>\r\n<div>\r\n	P = prevalence of event, is estimated&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; to be at 81% &nbsp;(Barata et al. 2012) &nbsp; &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Confidence interval = percentage of acceptable error</div>\r\n<div>\r\n	SS: 1.96 x 1.96 x 0.81 x0.19 &nbsp;= 59 images</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; 0.1 x 0.1 &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	A total of 48 patients were chosen on a voluntary basis during the data collection period. Two images were rejected by Radiologist for technical reasons (incomplete X-ray images) leaving 46 patients and 75 anatomical sites with comparison images (Figure 2).&nbsp;</div>\r\n<div>\r\n	The demography of the patients was listed in Table 1, 2, and 3. &nbsp;A majority of &nbsp;that (69.6%) were males, with a male female ratio of 2:1. The Malays made up 65.2% of studied patients, followed by Chinese patients (19.6%), Indians (6.5%), foreigners (6.5%) and other Malaysian race (2%). &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; <span class="Apple-tab-span" style="white-space:pre"> </span>Regarding age, 58.7% were between 18 to 40 years, 28.3% were between 41 to 60 years, and 13% were more than 60 years of age. Out of the anatomical sites analyzed, 69.3% were lower limb areas, and 21.2% represented upper limb areas. The patella was the commonest site imaged (24%), followed by the femur (17.3%). Chest (ribs) and clavicle made up 9.3% of anatomical areas studied (Table 4).&nbsp;</div>\r\n<div>\r\n	The kappa measure of agreement showed that agreement was moderate between ultrasound and X-ray (kappa of 0.41 to 0.6 was considered moderate agreement; above 0.80 was an almost perfect agreement). This mean that the agreement in the ultrasound diagnosis by the investigator and the X-ray diagnosis by the Radiologist was not merely due to chance. The table above shows that specificity of ultrasound is high for patella but sensitivity is lacking for this anatomical site. Due to irregularity of the patellar cortical outline when compared to the long bone surfaces, false positive findings of fracture occured. In femur ultrasound scanning, fractures proximal to the shaft, at the intertrochanteric or neck regions lay deeper and were more difficult to scan. This could lead to missed fractures of the proximal femur. The drawback of the patellar bone was its irregular appearance with cortical outline, and the femur was its proximal part positioned deeper in the &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;pelvis. Superficial long bones such as tibia, clavicle, hand and foot bones showed minimal to zero false positive or false negative findings. Ultrasound findings in these anatomical sites were more reliable. &nbsp;</div>\r\n<div>\r\n	Regarding the sensitivity and specificity of ultrasound in the detection of &nbsp;fractures, there was 72% sensitivity (95% confidence interval [CI], 50.6% - 87.9 %) and 80% specificity (95% CI, 66.3% - 90.0%), respectively, and the positive and negative predictive values were 64.3% positive predictive value (95% CI, 44.1% -81.4%) and negative predictive value 85.1% (95% CI, 71.7% - 93.8%), respectively. The positive likelihood ratio was 3.6 (95%CI, 1.96 &ndash; 6.6), and the negative likelihood ratio was 0.35 (0.184 &ndash; 0.666), with the odds ratio being 10.3 (95% CI 3.43 &ndash; 30.8).</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The triage area is the place in ED where the medical personnel assesses the severity of the patient&rsquo;s condition and allocates them to the appropriate zones of treatment. Triage also involves the &lsquo;see and treat&rsquo; concept where patients with mild disease conditions are given simple treatments and sent home (Cooke et al. 2003). A survey of adults attending the ED estimated that 13% of non urgent attenders could be directed away from the ED to self care (Coleman et al. 2001). The most frequent reason given by non urgent patients seeking care in the ED was their belief that X-rays were necessary (Coleman et al. 2001).</div>\r\n<div>\r\n	In terms of demography, the expected patterns were found. Patients presenting with suspected fractures were commonly males, usually in the age range of 20 to 40. This demographic group represented the more active members of society who were very productive, economically. Ability to quickly triage and discharge this group of patients will minimize disruption to their daily earning capacity. In terms of racial composition, the patient pool reflected the demography of the country with the Malay population forming a majority of the patients.</div>\r\n<div>\r\n	In the present study, the comparative accuracy of ultrasound versus X-ray in the triage environment was assessed. The use of mobile ultrasound, in addition to clinical decision rules and clinical judgment could reduce waiting time and eliminate the over utilization of X-rays in the ED. Out of the 75 images, ultrasound correctly identified fractures in 18 out of the total 25 identified with X-rays, and correctly ruled out fractures in 40 out of the total 50 identified with X-rays, thereby showing a sensitivity of 72% and specificity of 80%. The sensitivity and specificity of bedside ultrasound in this study were acceptable as compared to the gold standard of X-ray fracture diagnosis as reported by a Radiologist. A kappa inter-rater agreement found moderate agreement in the diagnosis of fracture by ultrasound in the ED as compared to fracture diagnosis using X-rays by Radiologist.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	In conclusion, the findings of this study showed that the bedside ultrasound is a viable tool to triage away non-critical patients who present to the ED. Further large multi-centre studies are needed to add on symptoms and clinical decision rules to bolster the accuracy of ultrasound findings in sending patient home. Patient and doctor perception of the ultrasound method, and time-cost analysis would strengthen the argument to replace X-rays with ultrasound to triage away non-critical patients with minor trauma in the future.</div>\r\n', 1),
(6059, 6050, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	FETOMATERNAL HAEMORRHAGE AND THE USE OF ANTI-D</div>\r\n<div>\r\n	Fetomaternal haemorrhage (FMH) may occur following a sensitizing event during pregnancy or at delivery and can lead to RhD immunization. If this occurs in a RhD-negative moman with a RhD-positive infant, there is a risk of HDN and can be fatal (Augustson et al. 2006). Assessment of FMH is an important element in determining the amount of anti-D to be administered to a RhD-negative mother following a sensitizing event or after delivery of an RhD-positive infant.</div>\r\n<div>\r\n	The guidelines for the use of anti-D immunoglobulin for Rh prophylaxis (National Blood Transfusion Services, Immunoglobulin Working Party, 1991) stated that at least 500 IU of anti-D must be given to every RhD-negative woman with no preformed anti-D within 72h of delivery of an RhD-positive infant. This dose will be sufficient to prevent sensitization from a 4ml fetal red blood cell bleed (BCSH Blood Transfusion and General Haematology Task Force 1999). Statistics have shown that 0.4% of women have a FMH of greater than 4 ml and up to 0.3% greater than 15 ml thus will not be protected by the standard 500 IU dose of anti-D (BCSH Blood Transfusion and General Haematology Task Force 2011; Bayliss et al. 1991). It is therefore important that the volume of any fetomaternal bleed is accurately estimated so that if necessary a supplementary dose of anti-D can be administered and maternal alloimmunization prevented.&nbsp;</div>\r\n<div>\r\n	Despite the policy, 1-2% of RhD-negative pregnant women at risk still become sensitize (BCSH Blood Transfusion and General Haematology Task Force 1999). A Kleihauer screening test is usually performed in all cases to detect the 1% of women who have a bleed larger than 4 mls. In these cases the Kleihauer test is used to quantify the haemorrhage and determine the additional dose of anti-D immunoglobulin. Cases with large bleeds are monitored for the disappearance of haemoglobin F containing cells using the Kleihauer test and for the presence of free anti-D in maternal serum.</div>\r\n<div>\r\n	Despite earlier publications describing methods for assessing fetomaternal haemorrhage (Duguid &amp; Bromilow 1999) it is recognized that there is little standardization of techniques (BCSH Blood Transfusion and General Haematology Task Force 1999; Johnson et al. 1993), a fact which can lead to inaccuracies in determining the size of any fetomaternal haemorrhage.</div>\r\n<div>\r\n	The Kleihauer acid elution test is the standard method for the detection and quantitation of fetomaternal haemorrhage in the UK. This test identifies fetal cells in the maternal circulation by the relative resistance of cells containing haemoglobin F to acid elution (Kleihauer et al. 1957). While the Kleihauer test is sensitive, its accuracy in quantifying FMH is open to question because of increased method and operator variability, low level of reproducibility, poor preparation technique and lack of standardization between laboratories and individuals.</div>\r\n<div>\r\n	Numerous sources of error includes variations in the thickness of blood films, the number of red cells in a low power microscope field, the fact that some fetal cells do not stain, and the difficulty of classifying cells of intermediate staining.&nbsp;</div>\r\n<div>\r\n	Furthermore, the Kleihauer test is unable to differentiate between fetal red cells and adult haemoglobin F containing cells which may lead to false positive diagnoses due to increased levels of maternal HbF cells as a result of physiological variations during pregnancy (25% in second trimester) or traits such as thalassaemia, sickle cell anaemia or hereditary persistence of fetal Hb (HPFH) (Duckett &amp; Constantine 1997; Corsetti et al. 1987). Descriptions of the Kleihauer test method vary between sources and therefore it is not surprising that wide variations between laboratories occur. In view of the problems with Kleihauer test, a new technology using flow cytometer for detection of HbF has been introduced.</div>\r\n<div>\r\n	Flow cytometry technique uses monoclonal or polyclonal antibodies to HbF, either directly conjugated with a fluorochrome or used with a secondary conjugated antibody to identify fetal cells in the maternal circulation (Davis et al. 1998; Brown &amp; Wittwer 2000). The red cells are fixed and permeabilized to allow the antibody access to the haemoglobin in the cytosol, and the intensity of the staining of the celIs distinguishes fetal cells from adult &lsquo;F&rsquo; cells. It allows large numbers of cells e.g. 50 000 - 100 000 to be readily counted. The test should then be more sensitive and accurate; and produce objective, quantitative results. However, flow cytometers are expensive and generally available only in major centers and require staff with specific expertise to operate the analyzer.&nbsp;</div>\r\n<div>\r\n	While the Kleihauer test is a simple, sensitive and cost effective technique for detecting patients with a FMH below 4 ml, flow cytometry offers several advantages in patients with larger FMH; 50 000 &ndash; 100 000 cells are assessed individually using the flow cytometer and several sources of inherent errors in the Kleihauer test are eliminated (Bromilow &amp; Duguid 1997; Duckett &amp; Constantine 1997; Corsetti et al. 1987). Raised maternal HbF concentrations render the Kleihauer test inconclusive, whereas the flow cytometer is able to distinguish fetal cells from adult cells. Furthermore, it can detect potentially immunising fetal cells even in cases where the Rh status of the fetus is not known. This method can be used to test mothers of any blood group and is not Rhesus dependent (Mollison 1972; Johnson et al. 1993; Nance et al. 1989).</div>\r\n<div>\r\n	OBJECTIVES</div>\r\n<div>\r\n	The objectives of this study were to determine the FMH using the flow cytometric analysis of anti-HbF antibody and to correlate the FMH using flow cytometric with anti-HbF method and the standard Kleihauer test.</div>\r\n<div>\r\n	MATERIALS &amp; METHODS</div>\r\n<div>\r\n	STUDY DESIGN</div>\r\n<div>\r\n	This was a comparative study to measure FMH in pregnant women using flow cytometry in comparison with conventional Kleihauer test. The study was performed using fresh whole blood collected in EDTA container from pregnant women attended the O&amp;G clinic or admitted in UKMMC between 2005-2006 with prior consent from respective patients before samples were withdrawn. All the samples were run in the flowcytometry laboratory, Blood Bank Unit, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Demographic and clinical data of these patients were retrieved from the Record Department of UKMMC. 98 pregnant women were recruited in this study. They were uneventful pregnant women from 28 weeks to 32 weeks period of gestation attending Antenatal Clinic (n=62), or pregnant women with sensitizing events after 20 weeks of gestation (n=15) or pregnant women after spontaneous vaginal delivery. Women who are known Thalassaemia were excluded. Three mls of maternal whole blood were collected into anticoagulated EDTA tube with prior consent. The samples were taken within two hours after spontaneous vaginal delivery, at any time during uneventful pregnancy between 28-32 weeks or whenever patient admitted to hospital after post-sensitizing event. The samples were mixed thoroughly and checked for haemolysis or blood clots. They were then sent to the laboratory and processed within four hours after collection. Haemolysed or clotted sample were rejected. The positive control sample was prepared in-house according to a documented protocol. The method described by the Members of the Scientific Subcommittee (SSC) of the Australian &amp; New Zealand Society of Blood Transfusion (ANZSBT).</div>\r\n<div>\r\n	Flowcytometry</div>\r\n<div>\r\n	In the flow cytometry method, the fetal red cells are identified using the monoclonal antibody against the HbF. The monoclonal anti-HbF used in this study was acquired commercially of Isotype IgG1, clone: WBAC HbF1 (Chemicon, USA). It was designed for the detection and quantitation of HbF positive RBC arising in the circulation of mothers as a consequence of fetomaternal haemorrhage (FMH). The format in the study used the purified immunoglobulin conjugated to FITC. SILENIUS&acirc;FITC conjugated anti-HbF was supplied in phosphate buffered saline, pH 7.2, containing 0.2% BSA and 0.1% Sodium Azide. To maintain its applicability, the monoclonal antibody was stored at 2-8˚C in undiluted aliquots and is protected from sunlight. The methods for the cell suspension and reagent preparations were performed and prepared according to the manufacturer&rsquo;s instruction. The reagents included the wash buffer, prefixation xolution, permeabilization solution and fixing solution. All the reagents were freshly prepared for every batch of test run. If not analyzed immediately, the cell suspension preparation can be stored at 4˚C in the dark for a maximum period of 24 hrs prior to analysis.</div>\r\n<div>\r\n	The cell suspension were then analyzed on a FAC Scan flow cytometer. For each test, 50,000 events gating were collected on the RBC population. A linear forward scatter versus linear side scatter gate was selected. The vertical scale of fluorescence intensity histograms were adjusted so that the small fetal-HbF region, the adult &lsquo;F&rsquo; cells and the adult negative cells could be visualized. The fetal-HbF region was set using the positive control and marked as &lsquo;M&rsquo;. The statistic analysis for the selected region was acquired and it showed the percentage fetal cells under the area marked as &lsquo;M&rsquo;. The FMH was then calculated using the formula according to BCSH guidelines on the estimation of FMH (BCSH Blood Transfusion and General Haematology Task Force 1999);</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	FMH (ml)=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Area under the curve (M) &nbsp;x &nbsp;2400</div>\r\n<div>\r\n	100 - Area under the curve (M)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	To optimize the flow cytometer settings, two following standards were used; Mouse anti-IgG antibody was used and run in parallel with each sample tested to identify any non-specific antibody binding. A positive control sample which contained 1% of HbF (10ml of cord blood added to 990ml blood of a male person) was run in parallel with each batch of samples tested.</div>\r\n<div>\r\n	Kelihauer&rsquo;s test (acid elution test)</div>\r\n<div>\r\n	The thin blood films were prepared from the EDTA-anticoagulated samples from pregnant women. It was done by an experienced senior laboratory technician in accordance with the laboratories standard method and according to the local haematology laboratory standard operating procedure. The positive control slide was made from the smear of fresh cord blood to differentiate between adult and fetal cells. The control slides were performed with each batch of slides and treated in exactly the same way as the samples. Prior to the quantification of the FMH, screening of the control slides was performed to ensure that the staining were satisfactory. If not, the whole process was repeated and fresh new slides were obtained for the same sample.</div>\r\n<div>\r\n	The quantification of FMH was carried out using a light microscope under 400 x magnification. An area of the film where cells were touching but not overlapping was selected. Any fetal cell was counted in the area. The slide was moved across in sequence so that the next counting was continued for a minimum of 6000 adult cells (Figure 2.10) ie. 15 fields examined under x400 magnification, and the number of fetal cells were recorded. The FMH was calculated using the formula:</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	FMH (ml) =&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Number of Fetal cell (F) &nbsp;x &nbsp;2400</div>\r\n<div>\r\n	Number of Adult cell (A)</div>\r\n<div>\r\n	Linearity study</div>\r\n<div>\r\n	Spiked samples were prepared by adding a known percentage of fetal cells to adult male cells (0.1%, 0.2%, 0.3%, 0.4%, 0.5%, 1%, 5%). They were subjected to the FMH quantification by both methods. The results of the tests are summarized in Table 1. This test represents the linearity study experiment on both methods.</div>\r\n<div>\r\n	RESULT</div>\r\n<div>\r\n	A total of 98 samples from pregnant women attended to Antenatal Clinic or admitted to UKMMC had quantification of FMH by flowcytometry with anti-HbF and Kleihauer test. The age of the patients are between 20 and 42 (median = 30). None of them have any background medical problem. The patients included women with uneventful pregnancy attending antenatal clinic (n=62), post spontaneous vaginal delivery (n=21) and women admitted for antepartum haemorrhage or other sensitizing events (n=15).</div>\r\n<div>\r\n	Linearity study</div>\r\n<div>\r\n	In the linearity experiment, the FMH estimated by the flow cytometer were plotted against the theoretical (known) estimated FMH (Figure 2). The results showed a correlation coefficient of r = 0.992. The FMH estimated by Kleihauer test plotted against the theoretical estimated FMH (Figure 3) showed a correlation coefficient of r = 0.87. The correlation study between the flow cytometer and the Kleihauer test for the FMH are shown in Figure 4. All the results of % fetal cells and the FMH were shown to be higher in the Kleihauer test compared to the theoretical estimation and the flow cytometry. The results of the linearity study showed a good correlation between the flow cytometry and the kleihauer test in estimating the FMH with the correlation coefficient of r = 0.896 respectively. All tests showed statistically significant results with p value less than 0.05 (p&lt;0.05)</div>\r\n<div>\r\n	Quantification of FMH in pregnant women</div>\r\n<div>\r\n	In this study, the results of the % fetal cells and the FMH from the patients&rsquo; samples showed fair correlation between flow cytometric method and Kleihauer test with correlation coefficient of r = 0.476 and r = 0.633 (p&lt;0.05), respectively (Figure 5 and 6). Overall, the results from the patients&rsquo; sample analysis of the FMH were shown to be higher by the flowcytometry in comparison to Kleihauer test. In 70 cases (71.4%), the size of FMH quantitated by flow cytometry were higher than that estimated by Kleihauer test. Only 27 cases (27.6%) showed that the FMH quantification by Kleihauer test were higher compared to flow cytometry. One case showed no fetal cell detected by both methods (1%). Twenty eight out of all cases (28.6%) were detected positive for fetal cells by flow cytometry method (0.06 to 5.5%) but was negative by the Kleihauer test. None of the results which were detected positive by the Kleihauer test were negative by the flow cytometer. These results, suggested that flow cytometer is more sensitive than the Kleihauer test in detecting the fetal cell. There were 23(23.7%) cases in which flow cytometry methods and the Kleihauer methods indicated a FMH of more than 4 ml and 45 (46.4%) cases indicated FMH of less than 4 ml, thus giving the concordance rate of 70.1%. A further 53 (54.6%) cases were associated with at least one method giving a result to suggest a bleed of more than 4 ml. Twenty one (21.6%) cases showed FMH more than 4 ml when estimated by flow cytometer method but less than 4 ml by Kleihauer test. Only 9(9.3%) cases with FMH more than 4 ml estimated by Kleihauer test were estimated less than 4 ml by the flow cytometer method. Analysis of all the results showed a fair correlation between the two methods for assessing the size of FMH and indicated that flow cytometry testing generally showed larger amount of FMH compared to Kleihauer test. However, flow cytometer seemed to be more sensitive in detecting the significant FMH of &gt; 4 ml.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	FMH estimation by flowcytometric technique can be done using monoclonal anti-HbF or monoclonal anti-D. The quantification by monoclonal anti-HbF has the advantage that it can be achieved irrespective of the blood groups of the mother and infants therefore enabling a flow cytometric method independently of the Rh antigens. Furthermore, the presence of maternal F cells did not interfere with the estimation since the intensity of staining is usually less than that of fetal cells. In the Kleihauer technique, there was no difference between an adult F cell and a true fetal cell. The addition of a calculated percentage of fetal cells from cord blood cells is used to help in identifying the cut-off level. Nevertheless, it is known that only 95% of fetal cells stain with Kleihauer and a small percentage do not. Flowcytometric method on the other hand differentiates the adult F cell and a true fetal cells by virtue of their intensity in staining of the HbF.</div>\r\n<div>\r\n	The current local policy for anti-D prophylaxis dosage is 500 IU (200 mg) of anti-D which can theoretically neutralize 4 ml of fetal cells. Additional dosage of anti-D is given according to the FMH estimated by the Kleihauer test. It is very crucial to estimate FMH accurately because underestimation of the FMH can be dangerous as a lower dosage of anti-D would be given to such cases therefore, would not be sufficient to clear the fetal cells from the maternal circulation. On the other hand, overestimation of the FMH may result in giving a higher dosage of anti-D to such cases and would not be cost-effective.&nbsp;</div>\r\n<div>\r\n	In our study, the linearity study results showed very good correlation between the known FMH and the results obtained by flowcytometric method and the kleihauer&rsquo;s test. There was also good correlation with the % fetal cells and FMH results obtained by flow cytometry and Kleihauer&rsquo;s test. However, all the results of the % fetal cells and the FMH from the Kleihauer test showed higher values than the flow cytometer and the estimated theoretical % fetal cells. The results of flow cytometric method on the other hand showed, concordance results to the estimated theoretical values. We therefore believe that the flow cytometer is a more accurate method to estimate the FMH than the Kleihauer test.</div>\r\n<div>\r\n	Duckett &amp; Constantine in (1997) had shown that a large inter-observer and inter-hospital variation in interpreting Kleihauer test slides where there was agreement in only 46% of cases. Janssen &amp; Hoffmann (2002) suggested that the quantification of fetal red cells by flow cytometry with monoclonal anti-HbF to be highly accurate based on their linearity study.</div>\r\n<div>\r\n	In our study, the results showed that there was a fair correlation between the % fetal cells and FMH results obtained by flow cytometry and Kleihauer test, r = 0.633 (p&lt;0.05). Previous studies done by different authors in observing the correlation between the two methods in quantitating the FMH have shown that there is good correlation between the two methods with the majority obtained a correlation coefficient r &gt; 0.8 (p&lt;0.05). Furthermore, our results, showed that in general, the flow cytometer gave higher results than the Kleihauer test. Other previous studies (Kennedy et al. 2003) on the other hand, showed that the Kleihauer test overestimates the FMH compared to the flow cytometry by using both anti-HbF and anti-D. However, they also found that in samples containing &lt;0.6% fetal cells, no significant difference in the detection of fetal cells between anti-HbF and anti-D (P = 0.11) but in samples containing &gt;1% fetal cells, anti HbF significantly underestimates the percentage of fetal cells (P = 0.0001). However, we were unable to determine from our findings whether the flow cytometry overestimate the FMH or as opposed whether the Kleihauer test underestimates the FMH. The conclusion may be determined by the use of another third outcome or a &lsquo;gold standard&rsquo; for the estimation of FMH. This &lsquo;gold standard&rsquo; has not been established yet. We also found in our study that some of the cases which were positive for fetal cells detected by flow cytometry were detected as negative by the Kleihauer test. None of the cases which were positive for the Kleihauer test were detected as negative by the flow cytometry method. These findings suggest that the flow cytometry is more sensitive in detecting the fetal cells.&nbsp;</div>\r\n<div>\r\n	The causes of the difference in the results from previous studies using flow cytometry have been suggested by some authors. The flow cytometry methodology with different reagents, the use of PE-conjugated anti-HbF instead of FITC-conjugated anti Hb-F, the use of PBS azide for washes and resuspension instead of PBS with bovine serum albumin, and a longer incubation time with the Triton-X reagent (8 mins instead of 3-5 mins). Another factor to be considered is the contamination by the white cell which can exhibit autofluorescence and may cause an overestimation of the FMH as demonstrated by Janssen &amp; Hoffman (2002). Several authors have suggested the use of monoclonal anti-CD45 or propidium iodide, a fluorescent dye specific for DNA to check for the contamination. Previous studies have mentioned various differences in the methodology that may cause the different results by flow cytometry. Johnson et al. (1995) and Llyod-Evans et al. (1999) have shown that monoclonal anti-D is more accurate compared with anti-HbF in detecting fetal cells and quantification of FMH. The use of phycoerythrin (PE) or fluorescein (FITC) as fluorescence dye may also cause different results. Few authors have suggested that the use of PBS azide rather than PBS with bovine serum albumin and longer incubation with Triton-X could contribute to the difference in the results. However, so far, there has been no individual study to look specifically into this matter. White cell autofluorescence can give false positive results. The use of double labeling with phycoerythrin conjugated anti-glycophorin A monoclonal antibody rather than single labeling with either anti-HbF or anti-D can improve the accuracy of FMH quantification (Llyod-Evans et al. 1999). The use of monoclonal anti-CD45 and propidium iodide to check for contamination of nucleated cells have also been suggested and proved to improve the accuracy (Janssen &amp; Hoffman 2002).</div>\r\n<div>\r\n	In our study, we used single labeling technique with FITC conjugated anti Hb-F, PBS azide during washing procedure and adopted &nbsp;the 3-5 minutes incubation time with Triton-X (permeabilizing solution) as recommended by the manufacturer. We did not employ double labeling to exclude white cell contamination. These different in the methodology may have contributed to our findings. We therefore, recommend to use a double labeling rather than single labeling of the monoclonal antibody in order to improve the accuracy in the quantification of FMH. We also would like to further evaluate and compare the use of monoclonal anti-HbF and monoclonal anti-D in the evaluation of FMH.&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, our study showed a fair correlation between the FMH quantification by flow cytometric method and the Kleihauer test. In addition, the linearity study showed that the flow cytometer is very accurate in detecting the fetal cells thus estimating the FMH.&nbsp;</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(6061, 6052, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Primary Low Grade Papillary Nasopharyngeal Adenocarcinoma has been scarcely reported in literature. It is a benign-like malignant neoplasm. The expertise of the Pathologist is required to identify specific histopathological features of this tumour. There are helpful immunohistochemistry markers for e.g. &nbsp;Thyroid Transcription factor-1 (TTF-1) which is expressed in subsets of such tumour, to aid with the diagnosis (Wu et al. 2007). An early diagnosis with a complete tumour removal offers a good prognosis with low risk of recurrence (Fu et al. 2008).</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 15-year-old boy presented with 5 months history of right nasal blockage and runny nose. There was no associated epistaxis. Upon examination, there was a mass at the centre of nasopharynx extending anterosuperiorly to the roof of right posterior choana. There was no evidence of cervical lymphadenopathy or anterior neck mass. Biopsy of the tumour showed Low Grade Papillary Nasopharyngeal Adenocarcinoma. Histopathology findings showed neoplastic tissue comprising papillary fronds and crowded glands lined by cuboidal to pseudostraitified columnar epithelium. Immunohistochemical studies revealed that these neoplastic cells were immunopositive for epithelial markers including Epithelial Membrane Antigen, Carcinoembryogenic Antigen and cytokeratin. They were negative for Thyroglobulin and S100.<span class="Apple-tab-span" style="white-space:pre"> </span></div>\r\n<div>\r\n	Paranasal sinuses scan obtained (Figure 1), demonstrated a well circumscribed 3 x 3cm polypoidal hypodense lesion arising from the roof of nasopharynx, obscuring the post nasal space. The lesion did not erode or destruct the base of skull (Figure 2). Subsequently, he underwent sphenodotomy, septectomy and removal of mass. Histopathology findings reconfirmed the result of LGPAC. Postoperative nasal endoscopies showed no evidence of mass in the nasopharynx and sphenoid cavities at 4 years, post surgery. Currently, the patient is on 6-monthly follow-up, and exhibited no clinical evidence of recurrent adenocarcinoma.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Nasopharyngeal carcinomas are established tumour classified by WHO into keratinizing or non-keratinizing carcinomas without evidence of glandular differentiation. The incidence of primary adenocarcinoma of nasopharynx is less than 0.3-0.4% of all nasopharyngeal malignancies (Fu et al. 2008). They can be divided into mucosal origin type and salivary type based on their morphological features. It is important to differentiate these two groups as the treatment and prognosis differ. Most of the mucosal type has papillary features and is low grade in nature. Low Grade Papillary Nasopharyngeal Adenocarcinoma (LGPAC) is categorized under mucosal type and to the best of our knowledge and limited reference available is extremely rare. &nbsp;It tends to arise in middle-aged group with female predilection (Ohe et al. 2010). As LGPAC is diagnosed in our patient who is a male teenager, it should be in the list of differential diagnosis in children or teenager with nasopharyngeal tumour.&nbsp;</div>\r\n<div>\r\n	Papillary nasopharyngeal adenocarcinomas can arise from 3 sources, which are mucosal surface, salivary gland or thyroid gland (as a form of metastasis). It is a known challenge to the Pathologists to propose a proper diagnosis as the histological features of malignancy from these origins are relatively similar to each other. LGPAC commonly involves the roof, lateral, or posterior walls of the nasopharynx and is described as exophytic with a polypoidal or nodular appearance. Majority of patients present with symptoms including nasal obstruction, epistaxis, aural fullness, tinnitus or otitis media. &nbsp;</div>\r\n<div>\r\n	Histologically, one of the significant findings is to observe a transition of the mucosal surface from normal epithelium to neoplastic surface. There are papillary projections lined by cylindrical to cuboidal stratified cells. There is no evidence of perineural, vascular or lymphatic invasion. &nbsp;It is important to carefully observe the histological findings of this tumour, as a subset of LGPAC can closely resemble metastatic papillary thyroid carcinoma (Wu et al. 2007).</div>\r\n<div>\r\n	Both have papillary architectures with fibrovascular cores, overlapping nuclei with clear chromatin, and psammoma bodies and this particular subset can express thyroid transcription factor-1 (TTF-1), thus gaining the acronym thyroid-like papillary nasopharyngeal adenocarcinoma. Therefore, when a positive HPE of LGPAC is obtained, a thyroid scan and systemic work-up should be carried out to rule out metastatic disease. Other helpful features to differentiate primary or metastatic LGPAC with positive TTF-1 include predominant stratified nuclei, absence of Tyroglobulin expression in tumour sections and negative thyroid imaging (Pineda-Daboin et al. 2006). Therefore, TTF-1 immunoreactivity in the absence of thyroid or pulmonary primary can be used as a diagnostic tool for LGPAC.</div>\r\n<div>\r\n	LGPAC acts relatively like a benign tumour. It has a low percentage of local recurrence and metastasis with no lymphatic spread being reported. The treatment of choice is complete surgical excision using transpalatal conservative techniques (Fu et al. 2008). Endoscopic excision is proposed for small tumour. Patients do not require subsequent radiotherapy as most of the cases are tumour free after a complete surgical excision but is beneficial for cases with incomplete resection to prevent recurrence (Fu et al. 2008).&nbsp;</div>\r\n<div>\r\n	Even though studies have reported such an excellent outcome with a very small possibility of recurrence, it is advisable to follow-up LGPAC patients regularly. Those with TTF-1 demonstrated, should have regular imaging study of thyroid gland as not to miss delayed presentation of primary thyroid malignancy (Wu et al. 2007).&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The rare papillary adenocarcinoma of nasopharynx requires experts knowledge in diagnosis, as an early identification may ensure good prognosis. Although, it acts in a benign manner, LGPAC should be of caution as one of a significant malignant tumour of nasopharynx.</div>\r\n', 1),
(6062, 6053, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Homicide followed by suicide is a rare incident which occurs with an incidence of 0.06/100,000 population, as reported in United Kingdom and Wales (Flynn et al. 2009) without sparing our country. However, the incidence of such an event cannot be determined, locally. On 11 September 2013, parents of three children were detained by police after their plan for filicide-suicide turned up to be a case of single homicide where only one life was claimed in the attempt. It was informed that the father had recent financial problems (Zolkepli 2013) with possibility of depression. This is to exemplify one of countless cases of homicide-suicide occurring worldwide. How could a depressed person decide to kill someone? Does homicide followed by suicide belongs to either one of the group (i.e. homicide or suicide) or it stands on its own category? In the present report, we relate a &nbsp;case of depression associated with homicide-suicide. The theoretical approaches to understand homicide-suicide phenomenon are briefly discussed.</div>\r\n<div>\r\n	Case Report</div>\r\n<div>\r\n	Mr A was a 30-year-old gentleman and single. He was admitted to Hospital Bahagia Ulu Kinta (HBUK) for alleged murder of a lady six months prior to admission. Before the incident took place, he was never diagnosed with any psychiatric disorder.&nbsp;</div>\r\n<div>\r\n	Retrospectively, he used to have few girlfriends and their courtships lasted for quite sometime ranging from 2 to 5 years. However, ultimately the relationships would end up unfavourably. Cheating due to extra-relationship affair and having one who was too difficult to manage due to time, these were the reasons given by him for the break-up with his previous girlfriends.&nbsp;</div>\r\n<div>\r\n	About one year before the incident, he met a woman who within four months of courting became his beloved one. He spent rather much in terms of time and wealth for that relationship. &nbsp;His savings sought from his five years job earnings were spent, if not all, for maintaining this relationship. &nbsp;Their relationship was described as perfect and smooth without any disturbance and it lasted for one year.&nbsp;</div>\r\n<div>\r\n	However, problems started to surface up when he suspected that she had someone else besides him. He self-investigated and with sufficient evidence had concluded that she was not loyal to him. He confronted her with the evidence he had but she denied it strongly.&nbsp;</div>\r\n<div>\r\n	That episode did not end up with a break-up but what was more heart-breaking was yet to come. A few weeks before the murder took place; his mind was very much distraught. He felt threatened with the impending end in the relationship. He felt useless and hopeless ruminating about his previous failures in relationships. His sleep was disturbed and his appetite was reduced. He was not able to focus and think clearly as he used to do. Furthermore, he found a few other sensible evidences to suggest the on-going unfaithfulness from the girlfriend&rsquo;s side (non-delusional). &nbsp;</div>\r\n<div>\r\n	Apart from this, he also suffered from stress at workplace which predated the relationship problem. He was not in good terms with his superior and he worked alone, literally, in his department. With this, he felt rejected not only by his girlfriend but also at work.</div>\r\n<div>\r\n	A day before the incident took place, he started to write a death note recounting his plan to kill his girlfriend and subsequently committing suicide. He while staying at his girlfriend&rsquo;s house left an important note to his family members with regard to his properties and wealth after his death. The next day, he carried out what he planned. Soon both of them were found unconscious and badly injured. However, he survived the suicidal attempt.&nbsp;</div>\r\n<div>\r\n	During admission his mental state examination revealed &nbsp;a young gentleman with psychomotor retardation. His speech was relevant but minimal with low tone and amount. His mood was low with suicidal ideation. There was no thought disorder and no perceptual disturbance. His cognitive and insight were good. He was diagnosed with major depressive disorder and treated with Escitalopram 20 mg daily.&nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Homicide-suicide can be defined as murder which is followed by perpetrator&rsquo;s suicide in one week time (Eliason 2009). However, this definition is far beyond unison (Liem 2010). The incidence of homicide-suicide case varies depending on regions but remains low around 0.05 &ndash; 0.22/100, 000 population (Panzcak et al. 2013). Due to various reasons, the psychiatric literature on homicide-suicide remains scanty. This is primarily because it is dealt mainly by non-Psychiatrist (i.e. police). In addition, both the perpetrator and victim are dead (Eliason 2009). Except in a few cases where the perpetrator is well-known or as in this case, the patient escaped from any potentially fatal injury.&nbsp;</div>\r\n<div>\r\n	Former views tend to view homicide-suicide as a variant of either homicide or suicide. However, in a recent systematic review and meta-analysis, the finding shows that homicide-suicide has a distinct characteristic differentiating it from suicide or homicide alone. It is often associated with &ldquo;intimate partner strain and general psychological stress&rdquo; (Panzcak et al. 2013).</div>\r\n<div>\r\n	Major depression, followed by substance abuse and psychosis are some of the commonest mental illnesses associated with homicide-suicide (Roma et al. 2012). In order to understand how depression leads to homicide-suicide, theorists have postulated numerous models aiming at understanding its manifestation.&nbsp;</div>\r\n<div>\r\n	Despite the different approaches employed by theorists to explain homicide-suicide, in general they share a common theme i.e. aggression as the underlying force of homicide-suicide (Liem 2010).</div>\r\n<div>\r\n	The theories of aggression can be subdivided into three subcategories in terms of its origin, direction and outcome as expounded by Liem (2010) (Figure 1). While the biological factor may not be apparent in this case, the source of aggression as a result of frustration is explainable from the strain theories point of view. Three types of strains have been described using Agnew&rsquo;s social stress and strain model to explain the source of aggression in homicide-suicide case. Any tension that prevents a person to achieve positively valued goal (i.e. sex, autonomy), removing or threatened removal of positively valued stimuli (i.e. loss of sexual partner) and real or threatened presentation of a noxious stimuli (i.e. rejection, abandonment) may lead to aggression (Harper &amp; Voigt 2007). However, this theory fails to explain the direction of the aggression either towards homicide or suicide. While attributional study can explain the direction of aggression (either suicide or homicide), however it cannot explain why after homicide the perpetrator proceed with suicide.&nbsp;</div>\r\n<div>\r\n	The end result of aggression that leads to homicide-suicide phenomenon can be explained from either psychodynamic or psychopathological perspectives (Liem 2010).</div>\r\n<div>\r\n	One of the earlier researcher in the field of homicide-suicide; Rosenbaum (1990) had the opinion that depression works as a defence to suppress the aggressive impulse. When a precipitating event, like a break-up in relationship which takes place, the defence would be broken and the aggressive impulse will be released (Rosenbaum 1990). Hendry and Short theorized that suicide is a result of strict superego, which prohibits an outward aggression. When a precipitating event take place i.e. break up, the defence is weakened and the impulse is directed outward as murder. Suicide may be undertaken as a form of self-punishment (Liem 2010).</div>\r\n<div>\r\n	As demonstrated in this case, patient&rsquo;s former experience of rejection and current involvement with work-related stress and impending rejection in relationship, had led to frustration and aggression. However, patient might have suppressed his anger which then manifested as depression. However as the stress went on uncontrolled and failed his defence which resulted in him acting out the aggression leading to homicide. Unable to tolerate made him decide to take his life away.</div>\r\n', 1),
(6066, 6057, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Charles Bonnet Syndrome (CBS) is a condition characterised by the occurrence of complex visual hallucinations which consists of formed images of objects or persons, predominantly observed in visually handicapped individuals and those with normal psychology especially in elderly. Its aetiology and pathogenesis are not well understood. CBS increases with age of the individual and ophthalmological diseases (Jacob et al. 2004). The prevalence also increases and cerebral diseases which includes age-related macular degeneration, cataract, glaucoma, cerebrovascular disorders and Alzheimer&rsquo;s disease (Rovner 2006). Estimates of prevalence among older adults range from 0.4% to 27% (Shiraishi et al. 2004). This case illustrates the presence of complex visual hallucinations of long standing duration in an elderly lady with visual impairment and later onset of cognitive impairment accompanied by other psychotic symptoms as well as the possible differential diagnoses that could be considered.&nbsp;</div>\r\n<div>\r\n	Case Report</div>\r\n<div>\r\n	An 81-year-old lady was brought by her daughter-in-law to Psychiatric Clinic, Universiti Kebangsaan Malaysia Medical Centre (UKMMC) with complaints of visual hallucinations for the past 18 months. Her visual hallucinations consisted of images of men, women, children, babies and inanimate objects. The images were smaller in size than normal (liliputian hallucinations). She was also noted to be talking to herself since past 6 months whereby patient claim she was communicating with the images that she visualised. She would go to the extent of preparing food and serving it to them and hosting the images that she visualised.&nbsp;</div>\r\n<div>\r\n	Her family members initially tolerated her behaviour but few months prior to clinic visit, patient started to visualise dead bodies in front of house and she insisted that her family members must perform prayers for the cremation ceremony. She would also wake up in the middle of the night, open the front door of her house and wait for the &lsquo;people&rsquo; she visualised in order to serve them food. She also had persecutory delusion towards her maid whereby she accused her maid of trying to seduce one of the images of men that she visualised. There was history of forgetfulness since past six months whereby patient frequently misplaced her belongings and sometimes had difficulty recognizing her own family members but it was not of utmost concern to the family members.&nbsp;</div>\r\n<div>\r\n	Patient denied having any depressive symptoms. There were no symptoms suggestive of mania. No history of alcohol consumption. There was no family history of psychiatric illness or dementia. She had no known medical illness. There was history of bilateral cataract surgery done 10 years ago. At that time, she had presented with blurring of vision of both eyes which was worsening over a period of 3 years. Premorbidly, patient was an introvert type of person, had few friends, hardworking and preferred to stay indoors spending time with her family.</div>\r\n<div>\r\n	Mental state examination revealed an elderly lady, moderately built and neatly dressed. She had poor eye contact, was not forthcoming and there was only superficial rapport established. She spoke in local Malay language. Her speech was of normal tone, rate and volume. Her speech was mostly coherent and relevant but it was irrelevant at times. Mood was euthymic and affect was congruent to thought. She had visual hallucination whereby she visualised images of people and inanimate objects. There was persecutory delusion towards her maid. She denied having any suicidal ideation. Cognitive assessment revealed that she was orientated to person but not to time, date and place. Her attention and concentration was poor. Her immediate, recent and remote memory was impaired. A Mini Mental State Examination score of 18 indicated moderate cognitive impairment. Her judgement was impaired and she had poor insight towards her illness.</div>\r\n<div>\r\n	Physical examinations were unremarkable. Ophthalmological examinations revealed reduction in bilateral visual acuity with measurement of 6/30 on right eye and 6/60 on left eye using Snellen chart. Neurological examinations were grossly normal. Her blood investigations were within normal range. Computerised Tomography (CT) scan of the brain was normal.</div>\r\n<div>\r\n	Patient was admitted for assessment and treatment. She was prescribed rivastigmine transdermal patch 5 mg once daily with olanzapine zydis 5 mg at night, which was then optimised to 10 mg at night a week later. Her behavioural and psychological symptoms showed improvement after two weeks of treatment. She also showed improvement in insight and activities of daily living on her subsequent follow-ups at the Psychogeriatric Outpatient Clinic.&nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The diagnosis made clinically based on DSM V was major neurocognitive disorder due to Alzheimer&rsquo;s disease with behavioural disturbance. It is evidenced by the presence of significant cognitive impairment which progressed gradually accompanied with behavioural and psychological symptoms of dementia (visual and auditory hallucination with persecutory delusion). Delirium was unlikely in this patient as she did not have fluctuation in her consciousness level. Another possible differential diagnosis worth considering could be very late onset schizophrenia.</div>\r\n<div>\r\n	Eventhough CBS is a less frequently diagnosed condition; it is a rather common cause of complex visual hallucination. CBS has even been reported in elderly patients without visual impairment (Arya 1995). Its prevalence in individuals with visual impairment varies from 10% to 15% (Menon et al. 2003). CBS occurs in 1.85-3.5% of psychogeriatric patients who have been referred to Psychiatrists by General Physicians, family Physician, and Ophthalmologists for visual hallucinations. CBS frequently goes undetected in clinical practice, due to both lack of awareness among doctors and patients&rsquo; reluctance to admit to hallucinatory experiences, for fear of being labelled as having mental disorder (Menon et al. 2003).&nbsp;</div>\r\n<div>\r\n	In this patient, the visual hallucinations were well formed, vivid and elaborate but pleasant to her, as described by de Morsier who stated that most of the patients found the experience to be pleasant (Finucane 2006). This patient had experienced CBS for duration of a year prior to the onset of neurocognitive disorder and emergence of other psychotic symptoms such as second person auditory hallucinations and persecutory delusions. These psychotic symptoms and cognitive impairment gradually improved with antipsychotics and anti-dementia drugs which made the diagnosis of major neurocognitive disorder with behavioural disturbances a likely provisional diagnosis. Hallucinations in Alzheimer&rsquo;s disease are more frequently visual than auditory, while delusions are typically of the paranoid, non-bizarre, and simple type (Bassiony &amp; Lyketsos 2003). A study by Pliskin et al indicated that patients diagnosed with CBS often manifest evidence of neuropsychological changes frequently associated with the early stages of dementia. Thus, CBS might be a predisposing factor to cognitive impairment, Alzheimer&rsquo;s disease or neurological deficit, or be an early marker of these conditions (Pliskin et al. 1996).&nbsp;</div>\r\n<div>\r\n	A differential diagnosis worth considering in this patient would be Schizophrenia (Very late Onset). The estimated one year prevalence rate of schizophrenia in individuals over 65 years of age is much lower than adults, between 0.1 to 0.5% (Copeland et al. 1998). Studies have shown that later onset is associated with a low prevalence of thought disorder and blunting of affect and a higher prevalence of visual hallucinations Hafner 1998). This patient had predominantly positive symptoms of schizophrenia such as visual and auditory hallucinations with persecutory delusions as well as cognitive impairment. However, in this patient, major neurocognitive disorder is a more likely diagnosis than very late onset schizophrenia as auditory hallucinations in the latter are more common than visual, delusions are more bizarre and complex, cognitive deficits are less severe, past histories of psychosis is common, and symptoms are more persistent, needing a long-term higher dose of antipsychotic interventions (Wetherell &amp; Jeste 2004) as compared to dementia with behavioural disturbances. This patient showed favourable response to low dose antipsychotic and anti-dementia drugs.&nbsp;</div>\r\n<div>\r\n	Even though several cases of CBS were reported in the literature, CBS is still often overlooked or misdiagnosed and hence under reported. Therefore, this case report highlighted the importance of CBS as a possible precursor to the development of major neurocognitive disorder, especially in the elderly population with impaired vision.</div>\r\n', 1),
(6073, 6064, '<p>\r\n	INTRODUCTION<br />\r\n	Malaria can be considered as one of the oldest and deadliest infectious diseases around the world. It continues to be one of the major public health problems in the world especially in African and South-East Asian countries (Kondrachine et al. 1997). As per the World Malaria Report, 2008, in terms of malaria incidence, India ranks 15th among 109 countries and contributed approximately 2% of global cases in 2006. India accounted for approximately two thirds of the confirmed cases reported in the South-East Asia region. Five states accounted for 60% of cases i.e. Orissa, Chhattisgarh, Madhya Pradesh, Jharkhand and West Bengal (Global Reports: World Health Organisation 2011). The state of Odisha, part of peninsular India is situated along the east coast extending from 170 49&rsquo; to 220 34&rsquo; N and 810 28&rsquo; to 870 29&rsquo; E. It is hyper-endemic for malaria. With 4% of India&rsquo;s population, the state contributed 28.6% of total malaria cases, 35% of P. falciparum cases and 50% of total malaria deaths in the country (Ranjit et al. 2005).<br />\r\n	Plasmodium falciparum causes one of the most life-threatening infections in humans. Despite all control measures, malaria still remains uncontrolled. Chemotherapy for malaria is the dominant mode of intervention since a vaccine for the disease is still not a reality. The most important factor is the development of resistance by P. falciparum to conventional and effective drugs like chloroquine, mefloquine, naturally occurring quinine and sulfadoxine/ pyrimethamine (Biot et al. 2006). Antimalarial drug development can follow several strategies, ranging from modifications of existing agents to design of novel agents that act against new targets. The rapid growth and multiplication rate of the malaria parasite during the intra erythrocytic life cycle with its metabolism intertwined with that of the host necessitates the parasite to acquire nutrients from the host cell. Purines enter the host erythrocyte via a combination of endogenous transporters and &ldquo;new permeation pathways&rdquo; induced in the erythrocyte membrane as the parasite matures (Gero et al. 1988; Gati et al. 1990; Upston et al. 1995; Quashie et al. 2010). The enclosure of Plasmodium falciparum in a parasitophorous vacuole inside the erythrocytes necessitates the uptake of nutrients or purines from host erythrocyte into P. falciparum across a multiplicity of membranes: the Red Blood Cell membrane (RBCM), the Parasitophorous Vacuolar Membrane (PVM) and the Parasite Plasma Membrane (PPM). A variety of complex and novel components may also be involved, including transporters, channels, ducts and tubulo vesicular membranes (TVM).<br />\r\n	One striking metabolic difference between all parasites and their mammalian hosts is the purine synthetic pathway. The mammalian cells are capable of synthesizing purine nucleotides de novo while the protozoan parasites studied to date including P. falciparum is incapable of making a purine ring (Shermann et al. 1979; Gero et al. 1990; World Health Organization 1997; World Health Organization Expert Committee on Malaria 2000). The enzymes of the salvage pathway for purine nucleotides have all been demonstrated in the infected erythrocyte. This purine salvage process is initiated by the translocation of purines across the parasite membranes. Thus, the transport characteristics of purine base and nucleoside transporters serve an indispensable nutritional function for the parasite, which may be a novel drug target (Carter et al. 2000).<br />\r\n	A P. falciparum encoded Equilibrative Nucleoside Transporter (ENT), the PF13_0252 (PfENT1) protein has been characterized in Xenopus oocytes and observed to transport purine and pyrimidine nucleosides and nucleobases (Carter et al. 2000; Martin et al. 2005). Functional analysis by genetic disruption in Plasmodium falciparum indicated that PfNT1 is essential for parasite survival under physiologic purine concentrations (El Bissati et al. 2008). It was also reported that PfNT1 knock-out parasites were unable to transport most physiologically relevant purine nucleosides/ nucleobases, although residual adenine transport was observed (El Bissati et al. 2008; El Bissati et al. 2006).<br />\r\n	The completion of the Plasmodium genome sequencing project (Gardner et al. 2002) also revealed three further ENT family members, PfNT2 (MAL8P1.32), PfNT3 (PF14_0662), and PfNT4 (PFA0160c). Like PfNT1, PfNT2, an intracellular permease is a member of the equilibrative nucleoside transporter family. Confocal and immunoelectron microscopic analyses of transgenic parasites harbouring green fluorescent protein- or haemagglutinin-tagged PfNT2 demonstrated endoplasmic reticulum localization. This study provided the first evidence of an intracellular purine permease in apicomplexan parasites and suggests a novel biological function for the parasite endoplasmic reticulum during malaria infection (Downie et al. 2010).<br />\r\n	The complexity of the intra erythrocytic environment where the parasite resides reveals that the location of PfNT3 and other putative members of P. falciparum ENT family are important because a restricted milieu can be a determinant of specialized functions and their potential as therapeutic targets. These may be novel sites of therapeutic targets. These predictions led to the importance of the study of putative nucleoside/nucleobase transporter, PfNT3 in in vitro cultured Plasmodium falciparum parasites propagated in human red blood cells. Such studies on PfNT3 are not yet reported in literature.<br />\r\n	MATERIALS &amp; METHODS</p>\r\n<p>\r\n	STUDY DESIGN<br />\r\n	Study of PfNT3 gene product by immunoblotting, immunofluorescence and immunoelectron microscopic localization was demonstrated in this work. This was based on in vitro culture of Plasmodium falciparum parasites, cloning, expression of the gene and polyclonal antibody development against the putative nucleoside/nucleobase transporter after approval of the Institutional Ethics Committee.<br />\r\n	PARASITE CULTURE AND ANALYSIS OF PFNT3<br />\r\n	RKL9 Clone of P. falciparum parasites (a strain of P. falciparum parasite in Rourkela, an endemic zone of malaria in the state of Odisha, India) obtained from National Institute of Malaria Research (NIMR), Delhi, India were propagated in human RBCs at 2% haematocrit and 1-10% parasitemia in RPMI-1640 medium (Himedia, India) supplemented with 10% heat inactivated human serum (O+ or AB+) (Trager et al. 1976).<br />\r\n	PfNT3 sequence was amplified by polymerase chain reaction with genomic DNA from the in vitro cultured P. falciparum parasites using a sense primer - 5&rsquo;- GCGTCATGAGTGACAGTCAGAG - 3&rsquo; (22 mer) and anti-sense primer - 5&rsquo;- GCTCGAGTTTTAAAATAACTGC - 3&rsquo; (24 mer), respectively. Optimized PCR conditions were initial denaturation at 980C for 2 mins., 30 cycles of denaturation at 980C for 30 secs., annealing at 45.70C for 30 secs., extension at 720C for 45 secs. and final extension at 720C for 10 mins. PCR product was identified by 1% agarose gel electrophoresis and sequencing. All the sequencing reactions were carried out with routine procedure of sequencing (commercially in an ABI 3130 genetic analyzer, USA at Manipal Life Sciences Centre, Manipal University, Karnataka, India). The PfNT3 PCR product in the excised gel was processed by QIAquick Gel Extraction Kit (Qiagen, Germany).Sequencing of the purified DNA was done in both directions. At least three independent clones were sequenced and each nucleotide was confirmed with an average of five times. Analysis of the sequenced PCR product and the deduced amino acid sequence was done by BLAST with the 3D7 strain (Plasmo DB database). The antigenicity and the hydophilicity property of the deduced amino acid sequence were found out (Hopp et al. 1981; Kyte et al. 1982).<br />\r\n	For study of identities, similarities and conserved regions of PfNT3, homology of the deduced PfNT3 amino acid sequence was searched by multiple sequence alignment (Clustal W2.0 program) after downloading different nucleoside transporter homologues from NCBI. The phylogenetic relationship was observed with PhyloDraw ver. 0.8. Topology was studied to predict the number of transmembane helices, positions of the N-terminal, the C-terminal and distribution of amino acids in different regions of the translated product (TMHMM software).<br />\r\n	PFNT3 ANTIBODIES<br />\r\n	A 108 bp fragment of the PfNT3 open reading frame encoding the 36 NH2-terminal amino acids of PfNT3 was amplified by polymerase chain reaction using a sense primer - 5&rsquo; - CGGGA TGGATCCATGAGTGACAGTCAG - 3&rsquo;, and anti-sense primer - 5&rsquo; - CAAAAGT ATCCTCGAGTTCAATATCACAAGG -3&rsquo; (Bangalore Genei, India) with engineered BamHI and XhoI restriction sites (strike through), respectively. The optimized PCR conditions were initial denaturation at 980C for 10 secs, annealing at 550C for 10 secs, extension at 720C for 10 secs and a final extension at 720C for 5 mins. The PCR product was identified by 1.5% agarose gel electrophoresis and sequencing.<br />\r\n	To generate an antigen for screening and purifying PfNT3-specific antibodies, the purified PCR product of PfNT3-1-108 sequence was cloned to pGEMT cloning vector, sub cloned into BamH1, Xho1 sites of pGEX-4T-1 (Amersham, USA) expression plasmid vector to create pGEX-4T-1-PfNT3-1-108 recombinant construct. Sequencing of cloned recombinant product was done to confirm proper ligation and reading frame. The recombinant construct was transformed into E. coli BL21 (DE 3) expression host for expression of the GST tagged fusion protein as described (Rabhi-Essafi et al. 2007). Expression was induced with 0.1 mM Isopropyl &szlig;-D-thiogalactoside. The expressed soluble recombinant fusion protein was analysed by SDS-PAGE and purified as per the New England Bio labs brochure. The NH2 terminal polypeptide of 36 amino acids was separated from the GST tagged recombinant protein by thrombin enzyme digestion and passage through the GST affinity column. The purified fusion protein was sent to Bangalore Genei, India for polyclonal antisera production. The antibodies were screened initially against the pre-immune serum and pGEX-4T-1-PfNT3-1-108 proteins by direct ELISA and Western blotting. Antibodies were purified using antigen immobilized agarose by Cyanogen Bromide method following manufacturer&rsquo;s protocol. Sensitivity and specificity of antibodies in the polyclonal sera was detected by ELISA and Western blotting. The pelleted parasites were lysed with Triton X-100 and sonicated gently for isolation of the parasite membrane fraction. Proteins were fractionated by 10% SDS PAGE.</p>\r\n<p>\r\n	LOCALIZATION</p>\r\n<p>\r\n	IMMUNOBLOTTING<br />\r\n	SDS PAGE fractionated membrane pellete, GST tag removed expressed protein, GST and preimmune serum were transferred to a polyvinylidene fluoride membrane (Millipore) by semidry blot method. The membrane was blocked with 5% milk powder (Millipore) in 1x TBS. Probing was done with purified developed primary antibody (dilution,1:1,000) and horseradish peroxidase conjugated Goat anti-rabbit IgG secondary antibody (dilution,1:20,000). A super signal west Pico Chemiluminescence kit (Thermo Scientific) was used to detect the proteins following manufacturer&rsquo;s protocol.<br />\r\n	IMMUNOFLUORESCENCE MICROSCOPY<br />\r\n	Slides for IFA were prepared as per standard procedure of slide preparation (Nicolle et al. 2001). In brief, RBCs harbouring an asynchronous culture of P. falciparum were washed twice in PBS, placed onto coverslips and dried at room temperature. Smears were fixed in ice cold acetone/methanol (70:30) at -200C for 5 mins, followed by rinsing 3 times in PBS and blocking in 2% (w/v) BSA. A PBS wash buffer containing 0.2% BSA, 2% goat serum was used for subsequent washes and incubations. Coverslips were incubated with purified PfNT3 antibodies (dilution: 1:50) for 4 h at room temperature. Controls included fixed parasites incubated with pre-immune antiserum. Smears were further treated with fluorescein isothiocyanate (FITC) conjugated secondary goat anti-primary antibody at 1: 300 dilutions for 1hr following the washing step. The cover slips were rinsed again with PBS washing buffer followed by incubation in 10 uM final concentration of Hoechst stain (Sigma) for 5 min. Mounting of cover slips on slides was done using vectashield mounting media (Vector Laboratories). Images were captured at intervals of 0.3&mu;uM along the Z axis using a Leica TCS SP5 AoBS Confocal microscope. Analysis of images was done by the LAS AF software version 2.0 at Centre for Cellular and Molecular Biology, Hyderabad, India.<br />\r\n	IMMUNOELECTRON MICROSCOPY<br />\r\n	Pelleted parasitized RBCs in a solution of 4% paraformaldehyde, 200 mM PIPES and 0.5 mM MgCl2, pH 7.0 were fixed on ice for one hour. The cell pellete was fixed for 30 minutes at room temperature in 10% gelatin after rinsing in PIPES buffer. Fixed samples were incubated overnight at 40C in 20% polyvinylpyrrolidone and 2.3 M sucrose in PIPES buffer. Samples were rapidly frozen in liquid ethane, cryosectioned and collected on carbon-Formvar-coated grids at Centre for Cellular and Molecular Biology, Hyderabad, India. Blocking was done with 5% foetal bovine serum, 5% goat serum in PIPES buffer followed by incubation in purified primary antibody (1:50 dilution) for 1hr at room temperature. Grids were rinsed, incubated with anti-rabbit secondary antibodies conjugated to 10 nm gold particles. Counterstaining was done with 0.3% uranyl acetate and 2% polyvinyl alcohol. Images were viewed on a 200 KV transmission electron microscope.<br />\r\n	RESULTS ANALYSIS OF PfNT3 AND THE TRANSLATIONAL PRODUCT<br />\r\n	The P. falciparum PfNT3 protein is a polypeptide of 437 amino acids encoded by a 1314 bp segment of DNA. Agarose gel eletrophoresis of amplified PCR product detected a 1.3 kb DNA (Figure 1) and approximately a 0.1kb band (Figure 2). The whole sequence analysed was significantly matching with the sequence of PfNT3 in PlasmoDB data base.<br />\r\n	CHARACTERIZATION OF ANTIGEN<br />\r\n	Hydrophobicity profile of the deduced amino acid sequence by Kyte and Doolittle hydropathy plot (data not shown) indicated PfNT3 to be a highly hydrophobic protein with 11 trans membrane helices. Predicted by homology study (Clustal W2.0 program, Figure 3), the deduced amino acid sequence was homologous to a number of ENTs, such as hENT1, hENT2, TbNT2, LdNT1.1, TgAT, PfENT1 and Fun26. T, D, P, R &amp; G amino acid residues were identical in particular regions of sequences but frequency of G was more. L, N, E, F, R &amp; S amino acids were observed as conserved substitutions with more frequency off. Semi conserved substitutions were also observed. More homology was found with nucleoside transporter homologues of PfENT1 and Fun26 (Figure 3). Obtained PfNT3 sequencewas more related to Yeast nucleoside transporter FUN26 and PfENT1 (Figure 4). Predicted topology analysis (TMHMM software) of the deduced amino acid sequence of PfNT3 presented an integral membrane protein with eleven trans membrane helices and an intracellular hydrophilic region between helices 6 and 7. It also revealed a cytoplasmic NH2 terminal of 49 amino acids and a short carboxyl terminal of 8 amino acids lying outside the membrane (Figure 5).<br />\r\n	CHARACTERIZATION OF ANTIGEN AND ANTIBODY SPECIFICTY<br />\r\n	The expressed GST tagged PfNT3-1-108 protein was of approximately 31.6 kDa as estimated from SDS-PAGE analysis (Figure 6). Purified polyclonal antibodies recognized a polypeptide with a molecular mass of 31.6 kDa (Figure 7-A). The single polypeptide in immunoblotting with thrombin digested GST tag removed expressed protein was recognized as a band of size 5.6 kDa in Figure 7-B. The purified antibodies recognized only a single polypeptide of approximately 50.9kDa on a Western blot parasite membrane lysate preparation (Figure 7-C, Lane-2). The parasite lysate did not reveal any band with GST used as negative control protein (Figure 7-C, Lane-1) and pre immune antiserum (Figure 7-D).<br />\r\n	IMMUNOFLUORESCENCE MICROSCOPY STUDY<br />\r\n	Immunofluorescence confocal microscopic images demonstrated the three dimensional distribution of PfNT3 in parasitized RBCs (Figure 8). Localization was in the region of the parasite as shown by the proximity of the parasite nuclear (Hoechst) stain (Figure 8, Panels 2, A-D) and dark brown haemozoin pigment in the bright field (Figure 8, Panels 1-A, and C, D). PfNT3 stained with FITC-conjugated secondary antibodies detected the protein in ring (Figure 8, panels B-3, 4), trophozoite (Figure 8, panels C-3, 4) and schizont (Figure 8, panels D-3, 4) stages of the parasite. All optical sections detected signals on the surfaces of intraerythrocytic ring, trophozoite and schizont stages of infected parasites in antibody treated slides (Figure 8, Panels 3, 4, B-D). It was neither expressed in erythrocyte cytosol nor in RBC membrane. A honeycomb staining pattern was observed in the mature schizonts (Figure 8, panels D-3, 4). There were no signals in all preimmune serum treated slides taken as control (Figure 8, Panels 1-4,A). A large number of cells examined showed similar results. IMMUNOELECTRON MICROSCOPY<br />\r\n	Further localization study was done by transmission electron microscopy using ultrathin cryosections of the parasites (Figure 9). Gold particles localized predominantly to the Parasite Plasma Membrane. No immunogold labelling was observed at the RBCM, TVM or any internal organelle including the endoplasmic reticulum (Figure 9).<br />\r\n	DISCUSSION<br />\r\n	Trans membrane transport is a critically important physiological process in all cells. Membrane transporters are responsible for the uptake of essential nutrients, modulations of concentrations of physiologically relevant chemicals. Parasite transporters are of great interest as drug targets. ENTs are a unique family of proteins with no apparent sequence homology to other types of transporters, which enable facilitated diffusion of nucleosides, nucleoside analogs used in chemotherapy across cell membranes (Hyde et al. 2001). Selective inhibition of parasite protein synthesis prevents the formation of the NPP suggesting that it originates from parasite proteins (Carter et al. 2000). With the goal of generating inhibitors specific for the target proteins of the parasite, several critical and unrelated biochemical pathways have been exploited for drug target identification (Cunha-Rodrigues et al. 2006).<br />\r\n	ANALYSIS OF PfNT3 AND THE TRANSLATIONAL PRODUCT<br />\r\n	The completely identical sequences of the PfNT3 and PfNT3-1-108 PCR products with sequences in Plasmo DB data base established the identity of the gene taken for study of PfNT3 nucleoside transporter protein.<br />\r\n	CHARACTERIZATION OF ANTIGEN<br />\r\n	A 108bp coding for polypeptide of 36 amino acids at the N-terminal of the protein was selected for cloning and expression because of highest local average hydrophilicity/antigenicity (Cunha-Rodrigues et al. 2006) and a hexapeptide is also the approximate size of an antigenic determinant (Hopp et al. 1981). The deduced amino acid sequence predicted homology but not complete identity with other nucleoside transporter homologues (Figure 3). Predicted PfNT3 sequences were specific as they were not completely matching to ENT sequences of other nucleoside transporter homologues (Figure 4). These results proved the expressed protein to be unique; validating it could be a site of novel drug target. Interpretation of Figure 5 is similar to the domain organization of ENT family members also.<br />\r\n	CHARACTERIZATION OF ANTIGEN AND ANTIBODY SPECIFICITY<br />\r\n	The expressed 31.6 kDa protein as estimated from SDS-PAGE analysis (Figure 6) was close to the calculated molecular mass of the encoded GST tagged fusion protein, the protein used as antigen. Recognized polypeptide with a molecular mass of 31.6 kDa in Figure 7-A corresponded with the PfNT31-108 GST tagged fusion protein size in the data base using ExPASy tool (www.expasy.ch, compute pI/MW). The revealed polypeptide of 5.6 kDa in immunoblotting with thrombin digested GST tag removed expressed protein in Figure 7-B also matched with the calculated molecular weight of the encoded protein (compute pI/MW). The polypeptide on Western blot parasite membrane lysate preparation (Figure 7-C, Lane-2) was also consistent with the molecular mass in the database. Specificity of the antibodies for PfNT3 epitopes was confirmed because the parasite lysate did not cross react with GST (Figure-7-C, Lane-1) and preimmune antiserum (Figure 7-D). These results proved that the antibodies specifically showed up the protein used for antibody development. These observations proved the presence of parasite genome encoded endogenously synthesized PfNT3 protein of parasite membrane origin in the P. falciparum parasites.<br />\r\n	IMMUNOFLUORESCENCE MICROSCOPY STUDY<br />\r\n	Presence of PfNT3 in parasitized RBCs was detected by reaction of the expressed protein with the antibodies (Figure 8). The honeycomb staining pattern observed in the mature schizonts (Figure 8, panels D-3, 4) was indicative of PPM staining of developing individual merozoites because the PVM circumscribes the whole collection of merozoites. This staining pattern was similar to the observation of PfNT1 protein (Nicolle et al. 2001) and the reticulated staining pattern of PfHT1 protein shown to be associated with PPM (Woodrow et al. 1999).<br />\r\n	IMMUNOELECTRON MICROSCOPY<br />\r\n	Immunoelectron microscopic study confirmed the membrane to which PfNT3 was localized. This was indicated by visualization of immunogold particles on the PPM (Figure 9).<br />\r\n	CONCLUSION<br />\r\n	Results of bioinformatics study on homology with ENT homologues, phylogeny and topology indicated PfNT3 probably to be a nucleoside transporter. This may have an important role in the survival of the parasite for being a route for translocation of the nucleosides/nucleobases, the vital nutrients essential for synthesis of the DNA and RNA of the parasite. Thus, it may have an important role in the growth and survival of the parasite. Immunoblotting, immunofluorescence and immunoelectron microscopy together established PfNT3 to be an integral membrane protein. PfNT3 appeared to be localized primarily to the PPM, although the possibility cannot be ruled out that it may be a minor constituent of other structures and uptake components of the complex nucleoside permeation pathway of P. falciparum. So, they may not only serve as routes for the targeting of purine analogs/cytotoxic agents into the intracellular parasite, but may also serve as antimalarial drug targets in their own right. Specific inhibitors can be designed against the transporter protein for inhibition of growth and survival of the malaria parasites. The parasite genome encoded protein being expressed in all the blood stages in human RBC can be prevented from expression by silencing of the gene by specific RNA interference or by gene editing (Crispr), thereby inhibiting the growth and survival of the parasite. With probable localization in the parasite plasma membrane the vital protein poses to be a novel molecular drug target for control of the lethal malaria caused by P. falciparum.<br />\r\n	Characterization of PfNT3 clone, a molecular component of complete nucleoside permeation pathway in Plasmodium falciparum may thus be validated as a new molecular chemotherapeutic target.</p>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(6078, 6069, '<p>\r\n	INTRODUCTION<br />\r\n	Tumour thrombus arising from advanced hepatocellular carcinoma (HCC) is not an infrequent complication. The usual sites of tumour thrombus are portal vein, hepatic vein, bile duct and inferior vena cava. Various treatment modalities have been proposed; including surgical resection, transarterial chemoembolization (TACE) (Fukuda et al. 2002), targeted molecular therapy (Sorafenib), arterial and systemic chemotherapy (Okada 2000), and radiotherapy (conformal radiotherapy (Igaki et al. 2008) and photon beam therapy (Mizumoto et al. 2007)). However, no single method has been proven to be effective in treating this complication (Mizumoto et al. 2007).<br />\r\n	The advancement of endovascular approach has now open new possibilities as a treatment option for various pathologies. Catheter directed mechanical thrombectomy is widely used in coronary artery disease, peripheral arterial disease and venous thrombosis (Sharifuddin et al. 2003; Cynamon et al. 2006). To our knowledge, endovascular approach to remove tumour thrombus has not been described previously.<br />\r\n	CASE REPORT<br />\r\n	A 85-year-old gentleman presented to the surgical out patient clinic with complaint of four months history of weight loss and vague upper abdominal pain. The pain was dull in nature and did not radiate to the back. Hemodynamically stable, his general physical examination revealed yellowish discolouration of sclera and mild cachexia. The abdomen was soft and non-tender with no mass palpable. Liver function tests (LFTs) was noted to be deranged, with bilirubin 79 mg/dL, alanine aminotransferase (ALT) 164 IU/L and alkaline phosphatase (ALP) 1120 IU/L. Ultrasound abdomen showed dilated intrahepatic ducts and common bile duct secondary to external compression from the enlarged porta hepatis lymph nodes. A large, irregular and solid lesion was noted to occupy the left lobe of liver almost entirely. Endoscopic retrograde pancreatography (ERCP) and endobiliary stenting was performed to relieve the biliary obstruction. Hepatitis screen was negative but alpha fetoprotein (AFP) was raised at 3520 ng/mL. A four phase computed tomography (CT) scan of the abdomen confirmed the presence of an ill-defined heterogenous liver lesion measuring 11cm (width) X 7cm (anterior-posterior) X 7.2cm (cauda-cranial), occupying segment II, III and Iva. The lesion demonstrated arterial enhancement and portal as well as delayed phase washout of contrast material. Thrombosis from the left hepatic vein was noted to extend into the inferior vena cava. The liver function test improved post biliary stenting with near normal ALP, ALT and bilirubin levels.<br />\r\n	A diagnosis of hepatocellular carcinoma with inferior vena cava thrombosis was made. However, surgical resection of the left lobe of liver was deemed inappropriate, given the extent of the tumour, patient&rsquo;s age and co-morbidities. A multi-disciplinary decision was made to treat this patient by means of transarterial chemoembolisation (TACE) and IVC tumour thrombectomy via endovascular approach.<br />\r\n	TACE was performed using a mixture of 50 mg Doxorubicin, Lipiodol and drug eluding beads. No immediate complication was observed. Almost three days later, he started to experience pain over the eipigastrium and left hypochondrium region. An urgent CT scan of the abdomen revealed multiple air locules within the infarcted liver tumor, suggestive of abscess formation post TACE. The liver abscess was successfully drained percutaneously under ultrasonic guidance and intravenous antibiotics commenced based on culture and sensitivity report. Endovascular thrombectomy of the IVC tumor thrombus was performed 3 days later. We highlight a novel method of IVC thrombectomy using AngioJet&reg; rheolytic thrombectomy system.<br />\r\n	AngioJet&reg; rheolytic thrombectomy system consists of an Xpeedior cathether, a pump set, and a drive unit. A larger 6F Expeedior is used to accommodate the larger lumen of an IVC. The pump set and drive unit creates a low-pressure zone at the tip of the cathether. This is done by emitting a jet of high velocity saline solution directed backward from the tip of the device outflow channels. The low-pressure environment causes fragmentation, and clots are aspirated through the effluent lumen (Cynamon et al. 2006). The pump operates at 8500 psi, and the speed of the jet can reach up to 450km/hr producing a vacuum effect of -600mmHg.<br />\r\n	The right femoral vein was punctured under aseptic condition and a 5Fr pigtail catheter was passed into the inferior vena cava below the liver level for a diagnostic angiographic run. Using an 8F guider and size 0.038Fr Roadrunner guide wire, a 6Fr AngioJet&reg; catheter was maneuvered to the thrombosed segment of the IVC. Series of thrombosuctions of 5 seconds were performed to a total time of 10 minutes. The patient developed macroscopic haemaglobinuria post procedure, which resolved spontaneously after 36 hours.<br />\r\n	RESULTS<br />\r\n	A 50% reduction of filling defect was seen immediately post thrombectomy. He was discharged well five days following the endovascular procedure. A repeat CT scan at three months showed no further propagation of IVC tumor thrombus. However, he succumbed to community-acquired pneumonia five months after the initial treatment of advance hepatocellular carcinoma.<br />\r\n	DISCUSSION<br />\r\n	HCC with IVC tumour thrombosis carry an inferior prognosis. If untreated, mortality is almost certain at 2-3 months from the time of diagnosis (Nagasue et al. 1984). Transarterial chemoembolization (TACE) and intravenous chemotherapy have marginal effect on survival (Bismuth et al. 1992). Surgical resection (hepatectomy with thrombectomy), combined with adjuvant hepatic artery infusion chemotherapy, is the more effective treatment option, although it only improves the survival to 7-8 months (Fukuda et al. 2002). In cases of tumour thrombus extending into the right atrium, there have been reported cases of successful hepatectomy with cava-atrial thrombectomy. However, the long term survival outcomes were not discussed (Ohwada et al. 2008; Leo et al. 2010). Catheter directed thrombolysis infusion (CDTI) has also been attempted but with no overall success (Stambo et al. 2006).<br />\r\n	Recently, photon beam therapy and conformal radiotherapy has shown promising results as the treatment modality for HCC with IVC tumour thrombus. A small case series by Mizumato et al. (2007) showed survival of more than a year upon treatment with photon beam. Treatment with conformal radiotherapy offers reasonable outcome with a median survival of 5.6 months (Igaki et al. 2008).<br />\r\n	AngioJet&reg; device is one of the few mechanical thrombectomy devices available in the market. The others include the Oasis (Boston Scientific, Natick, MA, USA), Hydrolyser (Cordis Europe, Roden, Netherlands), and Clot Buster Amplatz Thrombectomy Device (ATD, Microvena, White Bear Lake, MN, USA). Amplatz thrombectomy device has been reported to remove thrombus up to 73% at caval level (Delomez et al. 2001). Hydrolyser device has been successfully used to recanalise thrombosed ileofemoral and IVC and removing thrombosis around IVC filters (Poon et al. 2002). However, to the best of our knowledge, removal of IVC tumour thrombus has never been described previously.<br />\r\n	In this patient, significant amount of thrombus was removed, although complete thrombectomy was not achieved. Total clearance of thrombus is very difficult in large vessels like the IVC, although Delomez et al. (2001) documented angiographic evidence of 100% removal in their case series.<br />\r\n	Endovascular thrmboectomy aside, localised thrombolysis is widely used as adjuvant therapy to improve overall venous patency (Sharifuddin et al. 2003). Localised Urokinase infusion has been showed to be effective in lysing benign IVC thrombosis (Angle et al. 1998). However, Stambo et al. (2006) to lyse tumour thrombus using Tenectaplase for 24 hours did not show any significant improvement.<br />\r\n	The consequences of IVC tumour thrombus include IVC obstruction syndrome. The symptoms of IVC obstruction include progressive ascites, scrotal and lower body oedema. These symptoms are devastating to a patient with end-stage cancer. The use of rheolytic thrombectomy has the potential to provide symptomatic relief to these patients. However, re-thrombosis of the IVC is common due to phlebitis and residual thrombus (Gu et al. 1997). Anticoagulant therapy would maintain patency in normal circumstances, but its role in the prevention of tumour re-thrombosis is not known.<br />\r\n	CONCLUSION<br />\r\n	Our experience demonstrates the feasibility of rheolytic thrombectomy for the removal of IVC tumor thrombus. Mechanical thrombectomy could reduce tumour load and established patency of the IVC. The effect on the overall mortality rate is yet to be explored. There is scope for further fine tuning of the procedure to achieve the desired effect.</p>\r\n', 1),
(6081, 6072, '<p>\r\n	INTRODUCTION<br />\r\n	The misdiagnosis of right iliac fossa pain is not uncommon despite the emergence of advance imaging modality such as computed tomography, ultrasonography and laparascopy (Flum et al. 2001). The most common misdiagnoses in a female patient diagnosed with appendicitis are urinary tract infection, rupture ovarian follicle, pelvic inflammatory disease and ectopic pregnancy (Rothrock et al. 1995). The rate of error in managing pain in the right lower quadrant in the female sex or advance age can approach up to 40 percent (Andersson et al. 1992). The present short case highlights and discusses the challenge in diagnosing right iliac fossa pain in a post-menopausal patient.<br />\r\n	CASE REPORT<br />\r\n	A 66-year-old lady, ten years post-menopause presented to the emergency room with a week history of progressively worsening right iliac fossa pain. The pain was continuous, dull with no radiation. She had no alteration in bowel habit and was tolerating well orally. She had no prior medical illness or surgical history. On examination, she was haemodynamically stable and apyrexial. There was a well demarcated, mobile and tender right iliac fossa mass 158measuring 10 x 5 cm on palpation.<br />\r\n	Laboratory test revealed elevated inflammatory markers with a white cell count of 16.6x109 and haemoglobin of 12.6g/dl. Bedside trans-abdominal ultrasound showed a 6 x 5 cm heterogeneous mass with absence of a clear capsule at the right iliac fossa. Transvaginal ultrasound by a qualified obstetrician was unable to demonstrate the right ovary despite a normal left ovary. Urgent computed tomography (CT) scan revealed a right iliac fossa mass with a central dense foci and surrounding mesenteric streakiness suggestive of a collection. Bilateral adnexal organs were unremarkable. A CT guided biopsy produced a brown faecal aspirate.<br />\r\n	The patient gave the consent for an exploratory laparotomy and right hemi-colectomy. Intraoperatively, there was a large appendicular abscess encased by the greater omentum, terminal ileum and sigmoid colon; mimicking a caecal tumour (Figure 1). Instead, an omentectomy and appendectomy was performed. Histological examination confirmed a 8.5 x 5.0 x 5.5 cm non-malignant appendicular abscess.<br />\r\n	DISCUSSION<br />\r\n	A palpable large right abdominal mass in an apyrexial post-menopausal woman is highly suspicious of a gynaecological tumour. The patient was subjected to a laparotomy as CT imaging was suggestive of a perforated appendix whilst biopsy was suggestive of cecal diverticulitis. Newell et al. (1929) reported the first giant appendix weighing one pound and six ounces, mistaken for an intestinal malignancy (Newell et al. 1929). Care should be taken to avoid spillage as appendix of similar large nature may be of an appendiceal cystadenocarcinoma mucocele (Hassan et al. 2013). Moreover, the presence of an appendix mass should always be examined further as a caecal carcinoma not uncommon past the middle age (Hossian 1962).<br />\r\n	Although CT imaging has been proven to be a robust modality in diagnosing inflammatory appendix mass, it was not a conclusive diagnostic tool for this patient (Martin et al. 2015). In conclusion, an appendicular abscess, with minimal clinical evidence of on-going infection should not be rule out albeit its rarity in a post-menopausal women in her sixth decade. If clinical and imaging assessments are inconclusive, an exploratory laparotomy for a surgical excision is warranted primarily if malignancy is suspected.<br />\r\n	ACKNOWLEDGEMENTS<br />\r\n	We would like to thank all the staff from the Department of Surgery and Professor Dr. Srijit Das from the Department of Anatomy, Universiti Kebangsaan Malaysia for technical help.</p>\r\n', 1),
(6087, 6078, '<p>\r\n	INTRODUCTION<br />\r\n	Obesity, which is a chronic disease associated with comorbidities, should be controlled by physical exercise and changes in dietary habits, and surgical treatment is indicated when clinical measures are inefficient (Campos et al. 2007). Laparoscopic sleeve gastrectomy is a relatively new and evolving surgical technique indicated in the treatment of morbid obesity. This procedure has been gaining widespread popularity among both patients and surgeons. (Abraham et al. 2012a). Post-operative complications are mainly represented by gastric fistula, with an occurrence rate of 0-5.1% in the literature. This complication is difficult to manage and requires multiple radiological, endoscopic and surgical procedures (Burgos et al. 2009).<br />\r\n	CASE REPORT<br />\r\n	A 42-year-old male patient with morbid obesity underwent a laparoscopic sleeve gastrectomy in our hospital. During the post-operative course, the patient experienced abdominal pain and severegastro-oesophageal reflux symptoms. An upper gastrointestinal scope was performed, which showed fundal pouch lesion. Histopathological examination showed chronic gastritis for which he was started on esomeprazole (a proton-pump inhibitor). Two months later, he underwent a repeat upper gastrointestinal (UGI) endoscopy, which showed fundal pouch lesion covered with bubble of air. An abdominal computed tomography with oral contrast study was performed, showing collection of air under the diaphragm at left hemi-diaphraghmatic and peri-splenic area, which confirmed a leak. Drainage of the collection was performed and a bariatric giant stent was inserted, which covered the lesion.<br />\r\n	Six weeks later, the stent was removed and the patient recovered fully. A UGI study was performed, which confirmed absence of leak. The following figures demonstrate the procedures and findings as outlined above.</p>\r\n<p>\r\n	DISCUSSION<br />\r\n	Gastric fistula following bariatric surgery occurs in about 3% of the cases, with increasing risk up to 8% in second operations, and most commonly occurs at the angle of His (Campos et al. 2007). It has been hypothesized that gastric fistulas and anastomotic leaks commonly occurs due to poor blood flow, causing ischemia to the gastric wall at the staple line (Abraham et al. 2012a).<br />\r\n	Laparoscopic sleeve gastrectomy is the new surgical method to tackle morbid obesity. It has shown to be an effective yet justifiable weight reduction method and is gaining enthusiasm among general populations. This surgery overcomes obesity via reduction of the gastric size and removes the fundal ghrelin. Thus inducing early satiety producing cells and also decreases appetite (Serra et al. 2007). Sleeve gastrectomy is not exhaustive from severe complications too, which includes bleeding of the staple line in nearly 2%, stricture of the mid-portion of the tubular stomach in 1%, and gastric leaks with incidence varying from 0.7 to 20% (Tan et al. 2010). Incidence tends to be higher intraining centres owing to less experienced surgeons to a higher complication risk. Radiologic contrast studies with barium sulphate are routinely performed in certain centres on the post-operative day 3 which increases the chance of detecting minute subclinical leaks not normally seen with a liquid contrast.<br />\r\n	Past research reports have classified leaks based on duration of appearance, clinical severity and location of leaks after surgery (Csendes et al. 2005). The period leaks appear could be groups as: early - between first and fourth day post-surgery; intermediate - between the fifth and ninth day after surgery, and late - appearing after day ten post-surgery. In terms of clinical severity, Type I or subclinical leaks are those that appear as localized without spillage, few clinical manifestations and suffice for medical treatment. Type II includes those with dissemination into the abdominal or pleural cavity. Leaks appear most commonly, with 85.7%, in the proximal third of the stomach, close to the gastro-oesophageal junction compared to only 14.3% in the distal third (Burgos et al. 2009).<br />\r\n	Most authors suggest that the well-known risk factor for gastric leaks is attributed to ischemia along the gastric wall adjacent to the staple line and not dehiscence of the staple line itself. Classic ischemic fistulas tend to surface between Day 5 and 6 post-surgery when wall-healing process is between inflammation and fibrotic phase. Mechanical fistulas are usually discovered earlier, within the first two days post-surgery (Marquez et al. 2010).<br />\r\n	Clinical spectrum of patients with gastric leak varies accordingly, from the complete asymptomatic patient to patients with sepsis, multi-organ failures and followed by death. Patients usually present with fever (81%), epigastric pain (69%), tachycardia (44%), and leukocytosis (75%) (Csendes et al. 2010). Methylene blue test, upper gastro-intestinal gastrografin study and computerized axial tomography test is sensitive and effective to detect potential leaks (Csendes et al. 2010).<br />\r\n	Treatment of fistulas and anastomotic leaks should be tailored to the clinical state of the patient. Haemodynamic instability patients without regards of sepsis require prompt surgical re-intervention. The time initial clinical symptoms, presentation to hospital and diagnosis, as well as the time for initiation of therapy are crucial and will help in the patient outcome. Early leaks usually require prompt surgical approach. On the contrary, intermediate and late leaks, in majority of cases, are managed conservatively (Csendes et al. 2010). Conservative or medical management includes introducing abdominal drains, parenteral or enteral nutrition, high-dose proton pump inhibitors, broad spectrum antibiotics, use of biological glues (such as Seamguard, Tissucol, and fibrin sealant), and use of flexible coated stents when necessary.<br />\r\n	Giant bariatric stentis starting to gain popularity with few recently published reports. There have been successful deployments of a covered stent in a series of three patients with no complications relating to the stent placement (Serra et al. 2007). The incorporation of coated self-expanding stents was successful in six patients with control of leaks in 83%. It was reported of a 50% success rate for closure of leaks with four patients requiring premature removal of the stent due to migration, hematemesis, or obstruction from kinking at the proximal aspect of the stent (Tan et al. 2010). Evidence had also revealed complete healing in all three patients treated with endoscopic insertion of bariatric gastric stents (Casella et al. 2009). Inbar et al. (2011) reported five patients with oesophago-gastric leaks or post-operative fistulas, in which self-expanding metal stents were successfully inserted and retrieved following full recovery of all patients.<br />\r\n	Several considerations should be made when using stents for the management of gastric leak post laparoscopic sleeve gastrectomies. Endoscopic assessment should be performed at initial stage to evaluate the site and size of leakage and also the viability of the conduit. Gastric leaks at the proximal and mid-aspect of the gastric sleeve are amenable to endoscopic treatment with a stent (Burgos et al. 2009; Tan et al. 2010). A leak at the distal staple line near to the gastric antrum would not be amenable via endoscopic stenting as the stent would be too small, thus unsuitable to provide appropriate sealing effect to the defect. The gastric sleeve diameter should also be evaluated at the time of endoscopy for the selection of the appropriate endoscopic stent size. A larger-sized stent would be less predisposed to migration (Csendes et al. 2010). Another strategy to minimize or preclude stent migration would be using a longer stent whereby the distal aspect of the stent is rested along the wall of the gastric antrum (Inbar et al. 2011). Furthermore, any abdominal collection should be drained wither by laparoscopic or percutaneous means in combination with nothing per oral and nutritional support with either total parenteral nutrition or jejunostomy feeding (Serra et al. 2007; Csendes et al. 2010).<br />\r\n	Nausea, vomiting, drooling, early satiety, and retrosternal discomfort are the commonest symptoms after stent placement and tend to resolve within the initial days (Csendes et al. 2005). Stent migration is one of the main concern in a third of the patients. Stent migration warrants immediate stent replacement, removal, relocation, or passing out per rectum. Most authors recommend a period of 6-8 weeks as the optimal time to withdraw the stent. This can be challenging as adhesions are common and therefore might lead to mucosal tears and bleeding post-procedure (Marquez et al. 2010; Csendes et al. 2010). Few centres routinely perform methylene blue test at the end of the surgery. However, a negative test only demonstrates that the surgical technique was appropriate at that time (Csendes et al. 2010).<br />\r\n	Given the complexities in management of gastric leaks, it is likelyachievable those patients may have improved with solely conservative management (Casella et al. 2009; Inbar et al. 2011). The usage of stent could lead to a more predictable outcome and a speedier recovery and every effort must be made to prevent its occurrence (Csendes et al. 2010; Casella et al. 2009; Inbar et al. 2011). General surgical principles such as careful selection of patients, experience of the surgeons, gentle handling of tissues, appropriate surgical techniques including careful management of electro-cautery, and vessel sealing are essential.</p>\r\n<p>\r\n	CONCLUSION<br />\r\n	Gastrointestinal fistulae are a common occurrence after surgery, but surgical revision is associated with high morbidity. Patients with fistulae may already have concurrent infection, nutritional deficiency, or organ failure, making surgical revision more difficult. Despite the inherent challenges in this patient population, endoscopic therapies for fistula closure have demonstrated safety and efficacy. These techniques can be used alone but are even more effective when used in combination. Further research is on-going to determine which fistula types are most suitable for a specific endoscopic therapy. As evidence builds for the efficacy of these endoscopic techniques relative to surgical management, gastroenterologists will play a more prominent role in the treatment of gastrointestinal fistulae. Clinical suspicion is the hallmark of detecting gastro-peritoneal fistulas and anastomotic leaks. Confirmatory test is essential in aiding the diagnosis to help the detection rate to be better. Endoscopic assessment is essential for both diagnostic and therapeutic purposes, and could be an option of treatment of gastro-peritoneal fistulas in the future.</p>\r\n', 1),
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(7257, 7248, '<div>\r\n	Dear esteemed readers and authors,</div>\r\n<div>\r\n	Greetings from the desk of the Editor-in-Chief! At the very beginning, I wish to convey my sincere thanks to each one of you for the journal&rsquo;s growing popularity. It has been exactly 10 years of publication since the &lsquo;Medicine &amp; Health&rsquo; journal began its journey in 2006. Heartiest congratulations to everybody!</div>\r\n<div>\r\n	Over the last decade, many hurdles were encountered but we managed to sail smoothly. I am happy to inform you that the journal has started a new section on latest techniques. This section comprises articles which educate the medical personnel with latest techniques and methods which may be beneficial for better treatment.</div>\r\n<div>\r\n	On its 10th anniversary, I am happy to let you know that all research articles published in &lsquo;Medicine &amp; Health&rsquo; journal will have digital object identifier (DOI) number with immediate effect. I convey my sincere thanks and gratitude to Universiti Kebangsaan Malaysia Medical Centre management, especially Prof. Dato&rsquo; Dr Raymond Azman Ali (Ex-Dean), Prof. Dr. Ima Nirwana Soelaiman (Deputy Dean of Research &amp; Innovation), Prof. Dr. Zaleha Abdullah Mahdy (Present Dean) and Prof. Dato&rsquo; Dr. Roslan Abdul Shukor (Chair, UKM Press) for their constant support in helping us to have the DOI with effect from the 2016;6(1) issue.</div>\r\n<div>\r\n	I also wish to thank all the editorial board members and reviewers for their support and help. We march ahead to the future with our sincere efforts for indexation of the journal in higher academic databases. I look forward to a favorable respond from all readers and authors.</div>\r\n<div>\r\n	Regards,</div>\r\n<div>\r\n	Prof.Dr.Srijit Das</div>\r\n<div>\r\n	Editor-in-Chief</div>\r\n<div>\r\n	Medicine &amp; Health</div>\r\n', 1),
(7261, 7252, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	The unpredicted admission without any warning causes high level of stress and anxiety among family members (Al-Mutair et al. 2013). The experience in Intensive Care Unit (ICU) creates an intense emotional situation for both patients and their family members (Noor Siah et al. 2012). According to Verhaeghe et al. (2007), the hospitalization of a patient to Critical Care Unit could aggravate emotional shock, denial, anger, despair, guilt and fear of loss for the family members. Therefore, the family members require accurate and comprehensible information which leaves room for hope to reduce stress (Van Horn &amp; Kautz 2007). However, research has shown that the needs of family members having a relative in Critical Care Unit have always been unmet by healthcare provider (Khalaila 2013).</div>\r\n<div>\r\n	In Malaysia, family bonding is very strong among family members. Therefore, it is a normal scenario that the ICU corridor will be crowded with family members visiting their loved ones. During this admission time, family members want to ensure that their sick relatives received the best nursing care. Consequently, the family members also need attention from health care providers in terms of obtaining information, support, comfort, assurance and proximity to cope and adjust themselves to the situation (Noor Siah et al. 2012). Getting attention from health care providers may reduce the family&rsquo;s stress level and help in coping with the environment of critical care. A previous study by Stricker et al. (2007) emphasized that delivery of care would be more effective once family needs can be met, which include information rather than focusing on patients&rsquo; diagnoses alone.</div>\r\n<div>\r\n	Stress level of family members is high during the first few hours of the patient being admitted to Critical Care Unit (Leon &amp; Knapp 2008). Family members needs are overlooked, as the patient becomes a central care for the nurses and at this same time the family becomes anxious and fear for their loved one (Siddiqui et al. 2011). For family members who are struggling with a difficult situation such as financial anxiety and child care, these can lead to depression or post-traumatic stress disorder (Pochard et al. 2005; Paparrigopoulos et al. 2006). Unmet anxiety family members needs has potential to affect patient care through disregard for the treatment, distrust in health care providers and threat of lawsuit; moreover these feelings can be transferred to the patient (Browning &amp; Warren 2006).</div>\r\n<div>\r\n	Effective communication between families and health care provider increases satisfaction with patient&rsquo;s care (Soltner et al. 2009). However, doctors and nurses were reported to be difficult to access for clear, understandable, and honest information about the patients&rsquo; medical condition (Bailey et al. 2010). Effective, even adequate communication does not readily occur in the ICU. Most of the conflicts with the health care personnel during their family member&rsquo;s stay are related to inadequate communication (Heyland et al. 2002). For nearly 30 years, families have indicated the need for communication with and information from the healthcare team in the ICU. However, this need has not yet been successfully met (Hashim 2007).</div>\r\n<div>\r\n	MATERIALS AND METHODSPARTICIPANTS AND DATA COLLECTION</div>\r\n<div>\r\n	Ethics approval was obtained for the study from the Universiti Kebangsaan Malaysia Research and Committee. Project number: FF-2014-128. Permission to conduct the study was also granted by the Director of the hospital where the study took place.</div>\r\n<div>\r\n	This cross-sectional questionnaire survey was conducted in March to May 2014 at General Intensive Care Unit (GICU) and Coronary Care Unit (CCU) at one teaching hospital in Malaysia. A convenience sample was recruited from family members of critically ill patients institutionalized in ICU and CCU. For this cross sectional descriptive study, the sample size has been calculated using simplified formula by Taro Yamane (1967) who gave a total of 275. This power calculation was based on a total of 884 patients admitted to the GICU and CCU in January to December 2013. Due to time constraints, the data collection was conducted in a two month period. As there were 884 admissions from January to December 2013, on average, there were 74 admissions to GICU and CCU in a month. On this basis, it was estimated that 148 family members should be recruited for this study as the duration of the data collection was 2 months. All family members who were 18 years and above, understood or were able to communicate in English or Malay, stayed together with patient at the same house were eligible for inclusion. Those with family members and who had cognitive and mental impairment were excluded from the study.</div>\r\n<div>\r\n	MEASUREMENT TOOLS</div>\r\n<div>\r\n	Critical Care Family Needs Inventory (CCFNI) adopted from Norris and Grove (1986) were used. This questionnaire was a self-reported scale measuring the family&rsquo;s needs during patients&rsquo; admission to GICU and CCU. There are 5 domains of family members needs measured by CCFNI, which are: i) Information - cognitive and reflect the family&rsquo;s need for consistent and realistic information from appropriate health care provider about the patient&rsquo;s condition, prognosis, care and treatment that has been delivered to patient.;ii) Proximity - A family&rsquo;s need to be near and close, including freely access and unlimited visiting hours to their admitted family members; iii) Comfort - related to the comfortable hospital environments for the family members waiting to visit; iv) Support - availability of resources and support systems for family members to cope up with the admission of their relatives in critical care unit, and include needs of personal and physical comfort; and v) Assurance - family&rsquo;s need of hope, honest communication and caring behaviors of the staff to family members.</div>\r\n<div>\r\n	Information Using Likert scale, family members rated their needs from scale 1 (not important) to scale 4 (very important). To determine whether the family needs were met, 30 items from the Need Met Inventory (NMI) developed by Warren (1993) were used. Using Likert Scale, family members rated their needs were met or not from scale 1 (never met) to scale 4 (always met). Seven questions pertained to respondents&rsquo; demographic data include age, gender, relationship to patient, educational level and monthly income were also asked. The questionnaire was translated to Malay language and then back translated to English by an English teacher who had Teaching English as a Second Language (TESL) qualification. The Cronbach&rsquo;s test for the CCFNI instrument was 0.92; and 0.93 for the NMI instrument.</div>\r\n<div>\r\n	STATISTICAL ANALYSIS</div>\r\n<div>\r\n	Statistical analysis was performed using SPSS version 21.0. The level of family needs in Critical Care Unit, the needs of family members being met, and the relationship between socio-demographic data and level of family needs in Critical Care Unit were analyzed. Data are summarized as means, standard deviations or percentages, as appropriate. ANOVA tests were used to analyze the relationship between categorized variables of socio-demographic data; age, respondent&rsquo;s relationship with patient, education level and monthlyincome with family needs in Critical Care Units. Independent T test was used to identify the association between family needs and gender of respondents.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 109 (74%) family members completed the questionnaires. The respondents consisted of 48 (44%) male respondents and another 61 (56%) were females. The demographic data (Table 1) showed that majority of the respondents 35 (32.1%) were 18-29 years old, 27 (24.8%) were 40-49 years old, 20 (18.3%) for each age group of 30-39 years old and 50-59 years old, and for age group above than 60 years old, the percentage were 7 (6.4%). For the relationship variable, 41 (37.6%) were relatives of patient, children consisted of 33 (30.3%), of which 20 (18.3%) were spouses, and 15 (13.8%) were parents. The level of education was categorized into three groups; primary, secondaryand tertiary. About 61 (56.0%) were tertiary, 35 (32.1%) were secondary and 13 (11.9%) were primary. Most of the family members 37 (33.9%) earned their monthly income between RM 1600 &ndash; RM2500.</div>\r\n<div>\r\n	The major need of the family members of critically ill patients in a Critical Care Unit was found to be assurance. It would be because family members feel there were still hope of their loved one to be discharged to ward as well as the appropriate treatment was given by the healthcare professionals. Hence, helping in coping the anxiety level and build trust and confident in them. Comfort was rated to be the last need been met among the family members by health care professionals in Critical Care Unit at a tertiary hospital (Table 2). This may be due to less need on comfort subscales among family members during first few days of admission of their loved one in Critical Care Units.</div>\r\n<div>\r\n	Needs of family members being met (Figure 1), shows assurance was the most need been met by the health care professionals (M = 3.69, SD = 0.334). Proximity need was ranked second (M = 3.53, SD = 0.406), information need (M = 3.52, SD = 0.397), support need (M = 3.45, SD = 0.498) and the least need been met was comfort (M = 3.40, SD = 0.641). Relationship between socio-demographic data and subscales of family needs found that respondent&rsquo;s relationship with patient and proximity needs was statistically significant, indicating that the proximity was influenced by respondent&rsquo;s relationship with patient, F = 3.759, p = 0.013 (Table 3). The children showed that their attachment with the patient is high and they need to be close and nearer to the patient among other respondents&rsquo; relationship with patient (mean = 3.68).</div>\r\n<div>\r\n	The respondent&rsquo;s monthly income was statistically significant with the support (F = 4.172, p = 0.004) and comfort needs (F = 2.724, p = 0.033) indicating that the support and comfort need was influenced by respondent&rsquo;s monthly incomes. The relationship between comfort needs with gender found that female showed highest mean score compared to male which indicate that female need more comfort from health care providers during their loved one admission to the Critical Care Units (Table 4).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Family members expected that they would be informed on every patient&rsquo;s situation and progress. The details of patient&rsquo;s current condition and progress have been found to be the most information demand by family members. Findings of this study showed that Malaysian families are concerned when their relatives admitted to Critical Care Units. The principle of informing family members at home about changes in the patient&rsquo;s conditions by nurses are not a common practice in hospitals in Malaysia as they needed to be nearer to the ill person physically and emotionally (Hashim &amp; Hussin 2012). Therefore, great effort should be given by health care professionals especially critical care nurses to the family members. By doing so, it can build a trust and effective communication which can prevent most of the family members misunderstandings towards health care professionals. This is because the health care provider helps in build confident among the family members by assuring that their loved one was treated well and encourage the recovery phase. Lack in providing assurance to family members may result in anxiety and feeling of uncertainty of the survival rate of the patient (Hashim &amp; Hussin 2012).</div>\r\n<div>\r\n	The findings of this study also revealed that most of the family members highly focused on the care of the critically ill patient. With the high stress level and low coping mechanism with the critical situation, family members will burnout and became more emotional in which will affect their health status especially elderly family members (Yousefi et al. 2012). Nurses need to be sensitive of family members&rsquo; words and feelings to identified the problems and help them to cope with the situation. It is also important to empathize and provide adequate information by updating patient&rsquo;s condition from time to time to the family members which will help them to understand patient&rsquo;s condition. Critical care nurses should aware the importance of holistic approach in fulfilling family members needs of critically ill patient (Noor Siah et al. 2012).</div>\r\n<div>\r\n	The least need of family members of critically ill patients being met by the healthcare professionals was the comfort need, with the mean score of 3.40 and standard deviation of 0.641. The two questions being asked for this need are: &ldquo;to feel accepted by the hospital staff&rdquo;, and &ldquo;to have a telephone in waiting room&rdquo;. Few of the reasons of family needs were not met by the healthcare workers due to lack of time and knowledge, focus on the patient&rsquo;s need only, and not understanding the relationship between family members&rsquo; needs with patient outcomes and because healthcare workers do not recognize meeting the family needs as their conscientiousness (Hashim &amp; Hussin 2012). Besides that, the needs of comfort were least met as there was no telephone provided and there was no waiting room at the ICU and CCU for the family members of patient at UKMMC. It can be assumed that the telephone was not provided by UKMMC in the waiting room as all family members owned a cell phone. As the family members set their mind that the patient&rsquo;s welfare is the most important, the personal comforts of their own selves are no more a priority (Kosco &amp; Warren 2000).</div>\r\n<div>\r\n	The healthcare provider must also treat the family members as a human being including aspect of psychological and spiritual to help them cope with the condition of their loved one rather than just focusing to treat the patient. Thus, the family members could give the support to the patient to recover. The sudden unexpected unstable nature and strong probability of death, makes the families of patients become frailer. This are the time when nurses can play a role in giving support and comfort by listening to them or even acknowledged them as a human being. The second least need that being met was support need with a mean score of 3.45 and standard deviation of 0.498. Support can be given to the family members by the healthcare providers especially nurses through fulfilling the spiritual and psychological needs (McKinly &amp; Elliott 2008). Therefore, it is very important to instil the empathy sense in nurses and the nursing education of the importance of caring of the patient as well as the family members.</div>\r\n<div>\r\n	This study has found that only three family member&rsquo;s needs; proximity, support and comfort have relationship with socio-demographic data such as gender, relationship to patient and monthly income. There are significant relationship between respondent&rsquo;s relationship with patient and proximity need (F = 3.759, p = 0.013). Some of them expressed disappointment because could not visit patient after the visiting hour after they reached hospital after visiting hour.The children showed that their attachment with the patient is high and they need to be close and nearer to the patient among other relationship with patient (mean = 3.68). Other study by Stricker et al. (2007) also found similar findings that prove the relationship between proximity need and respondent&rsquo;s relationship with patient</div>\r\n<div>\r\n	Between the two gender, female showed highest mean score indicate that they need more comfort due to high stress level and lack of coping level to the critical care admission, hence their responsibility to other family members as well. They also tend to be more emotional and expect acceptance from the health care professionals (Wall et al. 2007). The respondents with lower income (less than RM 1500) suggest highly demanding for comfort and support from health care professionals during their loved one&rsquo;s admission in Critical Care Units.</div>\r\n<div>\r\n	IMPLICATIONS FOR PRACTICE</div>\r\n<div>\r\n	Nurses play an important role in fulfilling the family members&rsquo; needs in Critical Care Unit apart from doctor&rsquo;s involvement in updating patient&rsquo;s progress to the family members. A change in nursing administration entails nurses to evaluate family needs in a more universal manner. A family support group program could be developed with collaboration of various health care professionals. Nurses should pay more attention to the needs which will help to reduce the anxiety level and burden of the family members especially in the physical and psychological aspects. Nurses need to be prepared through continuing education programs towards sustaining a good collaboration with family members. This program will enhance the knowledge of nursing students in the care of patient and family members in crisis situation in a more holistic manner. This program should include the importance and priority needs of family members of critically ill patient such as effective communication, counseling and supportive nursing in crisis situation to effectively interact with the families and reduce the stress level of family members.</div>\r\n<div>\r\n	The researchers were unable to reach the targeted sample size of 275 family members of critically ill patient in the Critical Care Unit at UKMMC due to time constraint and relatives&rsquo; lack of cooperation especially spouse of the patient due to emotional stress. The achieved sample size was only 109 (74%) out of the actual sample size being calculated. Besides that, some of the relative refused to take part in answering the questionnaire due to language barrier especially Chinese family members. Future research is suggested with a longer period of data collection, larger sample and multi-language questionnaire. A qualitative approach to study the needs of the family members of critically ill patient might yield more valuable outcomes as a quantitative research can be limited by biasness of the family members input in answering the questionnaire. Valuable outcomes from the family members will be more helpful to improve in fulfilling the needs by the health care provider in critical care setting.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Meeting the needs of the family members of critically ill patient in Critical Care Unit at a tertiary hospital is an important aspect for a better health care delivery. It is important to identify the needs of the family members that have been met by the health care provider during admission to Critical Care Unit. Nevertheless, to fulfill the family members needs is not only an individual responsibility of critical care professionals, but should be assumed together with health institution managers. Fulfilling the needs of the family members in Critical Care Unit will be indirectly contributed in patient&rsquo;s recovery.</div>\r\n', 1);
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(7268, 7259, '<p>\r\n	Midazolam is a water-soluble imidazobenzodiazepine derivative which is commonly used for sedation in the Emergency Department (ED) (Ramoska et al. 1991). The reason attributed to this is that it possesses potent anxiolytic, amnesic, hypnotic and sedative properties (Khanderia &amp; Pandit 1987; Olkkola &amp; Ahonen 2008; Barash et al. 2009). The present study was to assess the incidence of adverse effects following intravenous midazolam given to patients in the ED. Reasons for sedation in the ED are chemical restrains of violent patients and procedural sedation and analgesia (PSA). PSA is the administration of sedatives to induce a depressed level of consciousness while maintaining cardio respiratory functions. This is done so that a medical procedure can be done (Godwin et al. 2005).&nbsp;<span style="font-size: 12px; line-height: normal;">10Procedural sedation is commonly used for outpatient medical procedures and done by non-anesthesiology physicians (Knape et al. 2007).</span></p>\r\n<div>\r\n	The use of midazolam in the ED is unique in terms that the location possess different issues and risks to the physician. Among issues that is different to normal outpatient clinic is the occurrence which is unpredictable, the timing is time dependent and sometimes emergent, gastric state is variable (either fasted or not). Patients are unselected and may have underlying systemic disease (Godwin et al. 2005).Hence, even though the adverse effects of midazolam was extensively studied in various clinical locations, the data for midazolam use in ED especially throughout Malaysia, is still lacking.</div>\r\n<div>\r\n	Adverse reactions from midazolam use include urinary retention, nausea, vomiting and excessive somnolence (Ramoska et al. 1991). Few serious incidents were reported with the administration of midazolam. In 1989, Roche Laboratories received 74 deaths reports with regard to midazolam use. Majority of the midazolam deaths were used as an adjunctive agent, which complicated with respiratory arrest. Overdosage, co-morbidities, old age and use in adjunct to other central nervous depressants are the main factors which lead to this mortality (Ramoska et al. 1991).</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a retrospective study conducted on 380 patients who received intravenous midazolam in ED, UKMMC from December 2012 to May 2014. Data collection commenced following receiving approval from UKMMC&rsquo;S Ethical Committee. Registration numbers of the patients that fulfilled all inclusion criteria were collected from the controlled drugs record. Data was then collected from the Records Department. Data was documented in the data collection sheet and then analyzed using Statistical Package for Social Sciences (SPSS) Windows Version 20.0. Chi-square test was used to analyze the association between the side effects of midazolam and factors studied. P value of &lt;0.05 was described as statistically significant.</div>\r\n<div>\r\n	The main objective of the was to look into the incidence of adverse effects after intravenous midazolam administration in the ED.</div>\r\n<div>\r\n	We also aimed to (i) determine the association between adverse effects of intravenous midazolam with socio-demographic factors (ii) determine the association between adverse effects of intravenous midazolam with co-morbidities and underlying illnesses (iii) compare adverse effects of midazolam when used alone with certain drug combinations (iv) determine the occurrence of life threatening adverse effects with intravenous midazolam administration.</div>\r\n<div>\r\n	SAMPLE SIZE CALCULATION</div>\r\n<div>\r\n	Formula by Cochran (1963),</div>\r\n<div>\r\n	n = x p (1 &ndash; p)</div>\r\n<div>\r\n	95% confidence level</div>\r\n<div>\r\n	z = degree of confidence value (1.96 for 95% confidence level) = 1.96</div>\r\n<div>\r\n	&Delta; = Error of margin 0.005 (0.5%)</div>\r\n<div>\r\n	p = expected prevalence</div>\r\n<div>\r\n	Based on an earlier study (Khanderia &amp; Pandit 1987), the overall complication of side effects midazolam was 1% and the error of margin 0.015</div>\r\n<div>\r\n	n = ( 1.96 )2 x 0.01(1 &ndash; 0.01)</div>\r\n<div>\r\n	0.01</div>\r\n<div>\r\n	= 380 patients (rounded to zero decimal points)</div>\r\n<div>\r\n	INCLUSION CRITERIA</div>\r\n<div>\r\n	Patients administrated intravenous midazolam in the ED, UKMMC from December 2012 to May 2014.</div>\r\n<div>\r\n	EXCLUSION CRITERIA</div>\r\n<div>\r\n	1. Midazolam indicated for rapid sequence intubation (RSI) &ndash; due to its use as an induction agent, not for procedural sedation and analgesia or chemical restrain of violent patients.</div>\r\n<div>\r\n	2. Patient transferred to another hospital.</div>\r\n<div>\r\n	3. Midazolam dependence.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	This study involved a total of 380 patients. The mean age was 42 years with minimum of one year and maximum of 108 years. Majority were males (64.7%) and Malays (43.2%) were the highest population involved. Other populations included Chinese (39.2%), Indians (9.2%) and others (8.4%). According to races, Chinese (5.0%) was the most population associated with side effects of midazolam use while Malays (43.2%) were the highest population involved in the midazolam use (Jabatan Perangkaan Malaysia 2010) (Table 1). For patients aged 40 years and below, the adverse effects were 6.3% (13 out of 206 patients), whereas for age 41-60 years, it was 11.5% (10 out of 87 patients) and 61-80 years were 10.6% (7 out of 66 patients). Incidence of side effects in patients more than 80 years is significantly higher (38/1%), (8 out of 21 patients (Table 1).</div>\r\n<div>\r\n	Two hundred and thirty nine (63%) patients had midazolam combined with other drugs. The drugs combined with midazolam were fentanyl (28.7%), morphine (16.6%), haloperidol (8.7%), propofol (6.1%), ketamine (2.1%) and diazepam (0.8%). Overall number of patients who suffered adverse effects recorded with midazolam administration was 35 (9.2%). Breakdown of overall adverse effects were excessive somnolence 10 (2.6%) patients, hypotension 7 (1.8%) patients, drug hypersensitivity reaction 6 (1.6%) patients, headache 5 (1.3%) patients and vomiting 4 (1.1%) patients and 3 (0.8%) patients had tachypnea, increased blood pressure and ECG changes.</div>\r\n<div>\r\n	Midazolam and fentanyl were the mostly combined drugs (47.4%) and it had the highest combination (4.2%) with side effects of midazolam use. Six patients who received this combination of drugs had transient hypotension. However, there was no significant association with p value of 0.38. Combination of midazolam with propofol or ketamine recorded no side effects. Table 2 showed the association of interaction of midazolam with one specific drugs and side effects whereas Table 3 showed the association of interaction of midazolam with various drugs combination and side effects. The total side effects with midazolam used alone was 20 (5%) compared to 15 (3.9%).</div>\r\n<div>\r\n	Among the comorbidities, hypertension (1.6%) was the highest comorbidity associated with midazolam use followed by asthma and diabetes mellitus which was 0.5% respectively. Other comorbidities were tabulated (Table 4). There were seven indications of midazolam and sedation (58.9%) was the highest indication of midazolam to be used. Other indications were orthopaedic procedures (17.9%), psychotic disturbances (10.5%), anticonvulsants (9.5%), anxiety (2.4%) and chest tube insertion (0.8%). Midazolam was given intravenously with dosage varying from 1.0 mg to 10.0 mg and the mean dose was 3.5 mg.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	We managed to gather a sample size of 380. The study population ethnicity breakdown comprised Malays-43.2%, Chinese-39.2%, Indians-9.2% and others-8.4% and it did not resemble the ethnicity of the Cheras region. This region has a population breakdown of 25.4% (3,101) Malays, 40% (4,910) Chinese, 7.6% (932) Indians and others (non-malay bumiputra, Malaysian nationals and foreigners) 27% (Jabatan Perangkaan Malaysia 2010). It resembles the population of Kuala Lumpur as a whole, which has a breakdown of 40.6% (644,406) Malays, 39.1% (621,805) Chinese and 9.3% (148,300) Indians. Although Chinese (5.0%) were more associated with side effects, there was no significant association between races and side effects of midazolam use (p=0.1). The highest gender associated with side effects was males (4.7%), and it correlated to the majority male patients who had midazolam administered. The correlation between gender and side effects was not significant (p=0.2)</div>\r\n<div>\r\n	Out of total 35 patients who experienced side effects, 13 patients (37% out of total with side effects) were below 40 years. Ten patients were between the age of 40 to 60 years and the rest (12 patients) were above 60 years old. There was no significance in the relation of age and adverse effects, as the distribution could be seen as nearly equal. However, when used in the extreme ages (above 80 years), a higher proportion of adverse effects were observed (38.1% - 8 out of 21 patients) as shown in Table 1. Elderly patients receiving midazolam 0.06 mg kg-1 showed significant reductions in mean blood pressure, respiratory rate and SpO2 (Sun et al. 2008). Elderly patients (&gt;70 years) previously on diazepam in particular has a higher propensity of falls compared to other benzodiazepam users (Ballokova et al. 2014). There was also an increase in fracture risk with short term benzodiazepine usage, (RR = 1.21; 95 % CI, 1.13&ndash;1.30) with a weak significance. This was also observed in the elderly age group (&gt;65 years) (Xing et al. 2013). Extra caution should be practiced when administering benzodiazepine among elderly, especially at extreme ages (&gt;80), as they are more susceptible to adverse effects. However, in the young (pediatric and adolescent), significant adverse effects are rare. A review by Papineni et al. (2014) showed that after oral administration of midazolam among the pediatric and adoloscents during dental procedure, the most common effect was paradoxical reaction, including aggressiveness.</div>\r\n<div>\r\n	There were five side effects which were encountered during the study and these were excessive somnolence, hypotension, headache, vomiting and allergy. However, none of them were life-threatening i.e. coma, respiratory arrest or cardiovascular effects. This study was comparable to the study by which reported no serious respiratory or cardiovascular events while using midazolam intravenously (Ramoska et al. 1991; Wenzel et al. 2002; Xing et al. 2013). Our study is similar to a previous study which included intravenous midazolam used for critical patients such as head injury, sepsis, meningitis and all types of drug combinations, which is sedative in nature (propofol and opioids) (Ramoska et al. 1991). Other neurological effects such as euphoria, aggressiveness, depression and intense hiccups were not documented in the clinical case notes, neither it was undetected nor classified as somnolence. As comparison to a study by Wenzel et al. (2002) neuropsychiatric effects including aggressive, euphoria and depressive behaviors made up to 6% of cardiac cases undergoing transesophageal echocardiography.</div>\r\n<div>\r\n	There were 6 (1.6%) cases reported on excessive somnolence involving midazolam alone and 4 (1.1%) cases reported with drug combination, which accounts to 10 cases (3.8%). Majority of excessive somnolence is due to repeated dosage given due to inadequate sedation from the initial dose. A 41-year-old male with clavicle and rib fracture was reported to have received total of 8 mg IV midazolam. The additional dosage was given due to his agitation at that time. He developed somnolence with duration of 2 hours with GCS 11/15. He was monitored later and did not develop respiratory or cardiovascular effects. The frequency is slightly higher than what was reported by (Ramoska et al. 1991) in which excessive somnolence was observed in (1.6%) of 120 patients.</div>\r\n<div>\r\n	The second side effect was 6 (1.6%) cases of drug hypersensitivity reaction. Two (1.1%) patients resulted dermatology effects with midazolam alone. Both cases developed itch and urticarial rash at a range of 1-2 hours after midazolam administration. The other 4 cases were in combination with other drugs i.e. opiates, hence cannot eliminate the combination or histamine release by opiods. The patient was stable afterwards after administration of hydrocortisone and piriton. Life-threatening laryngospasm or anaphylactic shock did not develop during the period of our study.</div>\r\n<div>\r\n	Hypotension was recorded in 7 (1.8%) cases. Patients with hypertension were found to have higher risk of developing hypotension with the administration of midazolam, which correlates with previous studies (Shekerdemian et al. 1997). Hypotension after midazolam administration was due to cardiovascular depressant effect that results in minimal change in arterial blood pressure, heart rate and systemic vascular resistance (Shekerdemian et al. 1997). Other contributing factors to hypotension were co-morbidity, other combination of drugs given and underlying illnesses. Another case reported was a 87-year-female who had hypertension, chronic kidney disease and complete heart block. She was given a 1.5 mg midazolam and 0.5 mg fentanyl for sedation. She developed hypotension which her blood pressure falls from 191/61 mmHg to 96/35 mmHg with heart rate of 36 beats per min after 4 hrs. Temporary pacemaker was inserted and later blood pressure was stabilized. In this case, hypotension developed might be due to several factors such as elderly age which affect the renal clearance and her clinical conditions like bradycardia and heart block. All cases of hypotension which developed post intravenous midazolam administration were transient and resolved with supportive measures.</div>\r\n<div>\r\n	Three (0.8%) patients developed vomiting. This is lower than previous reports (2-3%) (American Hospital Formulary Service 1989). Three (0.8%) patients developed headache post midazolam administration which then resolved. Adverse effects will be considered severe or life-threatening if it results in permanent organ damage or death. Adverse effects that are selected in this category are cardiac arrest, apnoea, anaphylaxis and severe respiratory depression which resultes in hypoxia and neurological sequelae. Papineni et al. (2014) classified life- threatening as prolonged respiratory depression and hypoxia. No case was reported with such serious side effects.</div>\r\n<div>\r\n	A limitation of this study was the retrospective data collection. We could not obtain all the medical files requested in a short duration. Few of the medical records provided were incomplete and inconsistent as there were no proper documentations of certain side effects from the files. Not all adverse effects were documented and are most neuropsychiatric effects were labeled under the group of altered mental state, or low GCS, whereas it could mean euphoria, aggressiveness or somnolence. It was also impossible to compare when drugs used in combination, whether the effects were due to midazolam alone, or confounding effect with other drugs. There was no significant result. This study was more towards a role of an audit, in which the adverse effects of midazolam in our ED did not differ much from the general population.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Statistical values showed that there was no significant association with the demographic factors and the drugs combination towards the side effects of midazolam use in ED. No life-threatening side effects occured following the use of midazolam, in consideration of the wide variety of critical patients. Thus, we conclude that midazolam is a very safe drug used&nbsp;in ED. In fact, special caution, close monitoring and precaution steps should be carried out during the administration of midazolam in ED. We should follow the guideline of management recommended in ED so that the usage of midazolam can be optimized with minimal side effects in clinical setting. Caution should be practiced in elderly. In addition, as in advent to introduction of new sedative agent such as propofol and etomidate, the role of midazolam is still considered to be safe.</div>\r\n', 1),
(7270, 7261, '<div>\r\n	Acute exacerbation of bronchial asthma (AEBA) contributes to a large number to attendance. In Sarawak General Hospital, there are about 700 - 800 cases per month, which translates to approximately 10% of total ED workload.</div>\r\n<div>\r\n	The rate of asthma admission has been constant throughout years especially among paediatric population, even though there is an increase in emergency asthma attendance (Russo et al. 1999). This is due even though attendance is increasing and many are not admitted due to high threshold for admissions. In developed countries, low asthma admission rates are an indicator for improved asthma care. Significant reductions in admission have occurred in several countries, such as North America, European countries and Australia (Anderson et al. 2008; Anderson et al. 2007).</div>\r\n<div>\r\n	Even though a large number of patients are discharged, a small percentage in developed countries revisited the ED. A study at Kansas City showed out of 4228 patients visited ED, 3276 was discharged and revisit within 7 days was 133 (4%). It was concluded that lower asthma quality of life scores correlated with frequency of revisit (Walsh-Kelly et al. 2008). This study aims to quantify the numbers of asthma revisits and identify influencing factors in our population. High percentage of revisit can translate to poor asthma care which increases the mortality and as factors that lead to this can be addressed.</div>\r\n<div>\r\n	The present study aimed to identify factors influencing early revisit of adult patients with AEBA discharged from ED. The specific objectives were: (i)to determine whether demographics (gender, age and ethnicity) are factors influencing early revisit (ii) to determine whether concurrent infection influence early revisit (iii) to determine if prescription of oral corticosteroid upon discharge influence early revisit (iv) to determine if the number of nebulizations given during the initial presentation influence early revisit.</div>\r\n<div>\r\n	MATERIALS &amp; METHODS</div>\r\n<div>\r\n	This was a retrospective cohort study.Data was collected from patient&rsquo;s medical charts who attended Emergency Department (ED) Sarawak General Hospital during March 2014. Study sample included all adult asthma exacerbated patients discharged from the ED of within the study period.</div>\r\n<div>\r\n	Ethical approval was obtained for clearance to access and use of patient data within the study period. It was registered under National Medical Research Register and approved by Medical Research and Ethics Committee under Universiti Kebangsaan Malaysia (UKM approval number UKM 1.5.3.5/244/FF-2015-225).</div>\r\n<div>\r\n	All adult asthma exacerbated patients discharged from the ED within the study period were included. Patients that were excluded were those who concurrently had other lung pathologies such as chronic obstructive pulmonary disease, acute pulmonary edema, tuberculosis and pneumonia. Revisit to ED for other diagnosis than exarcerbation of asthma was also excluded. Asthma revisits more than 2 weeks were also not included.</div>\r\n<div>\r\n	The case notes or triage forms of all adult acute exarcerbation of bronchial asthma (AEBA) cases within the study period were identified and collected from the records of Emergency and Trauma Department. Relevant data including demographics, case history, clinical findings, ED management and disposition from these cases were then transferred to the data collection sheets.</div>\r\n<div>\r\n	STATISTICAL ANALYSIS</div>\r\n<div>\r\n	Data entry and all analyses were performed using SPSS software version 12. Data was summarized using proportions, mean standard deviation and median. Univariate analyses of the relation of various factors to risk for ED revisit employed X2 test, Student&rsquo;s t test and Mann-Whitney test where appropriate. P &lt; 0.05 was considered statistically significant.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	The number of adult patients who fulfilled the study criteria was 397. Majority of patients who presented to the ED were Malays (59.2%) followed by Chinese (14.6%). Other ethnic groups like Iban and Bidayuh were 8.8% and 4.2%, respectively. Based on the clinical characteristics, majority of the patients were within the mild to moderate severity of asthma exacerbations. Mean heart rate was 100/min SpO2 pre-treatment was 98%. Divided according to sex, there were 178 male and 219 female patients (Table 1).</div>\r\n<div>\r\n	A total of 76.7% patients had concurrent infection during initial presentation to the ED. These included upper respiratory tract infections, tonsillitis, urinary tract infections and community acquired pneumonias. There were 99 patients (25.1%) who left the ED after receiving one to two nebulizations. Two weeks revisit rate was 13.9%, which contributed an extra ED workload of 15.2% .</div>\r\n<div>\r\n	Male gender had a higher percentage of ED revisit (56.4%), whereas female ED revisit was 43.6%. Out of the total 55 patients that revisited ED, 65.5% (n=39) had concurrent infection. Out of 342 patients who did not revisit ED, it was recorded that 78.7% (n=269) had concurrent infection.</div>\r\n<div>\r\n	Oral corticosteroid was prescribed to only 24.9% (n=99) of patients who initially attended ED for asthma exarcerbation. Of this, 10% (n=9) who were prescribed initially with corticosteroids, revisited ED. Eighteen percent (n=46) of patients who were not prescribed corticosteroids, revisited the ED.</div>\r\n<div>\r\n	Twenty five percent (n=99) of initial attendees to ED absconded before treatment could be completed. Thirty two percent (n=32) out of those who absconded during first presentation revisited ED, compared to 7.7% (n=23) of those who completed the initial treatment.</div>\r\n<div>\r\n	Although 42 patients revisited ED once within 2 weeks, only 4 of them were admitted. Of 2 patients who revisited ED 4 times, one of them was admitted. None of the revisits resulted in intensive care unit admission or hospital death.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This study obtained an ED revisit rate of 13.9% within two weeks of discharge. The results relate to previous studies done by Emerman et al. (1999) and Rowe et al. (2008) who reported revisit rates of 17% and 13.9%, respectively. In another study done by Yaacob et al. (1991) in Hospital Universiti Sains Malaysia reported a relapse rate of 18.8% within 3 months of discharge. Their study also looked at ED cases only with no telephone follow-up but instead of 2 weeks, they looked at revisits within a period of 3 months. Although, there is no common definition of an acceptable relapse or revisit rate, we think the finding of a revisit rate of 13.9% over short period of 2 weeks gives an impression of high treatment failure rate.</div>\r\n<div>\r\n	Causes of asthma relapse are multifactorial. Consistent with studies done by Emerman et al. (1999), patient&rsquo;s demographic data, including gender, age and ethnic group did not show any association with early revisit. Male patients had a slightly higher ED revisit (56.4%). However, it was not statistically significant.</div>\r\n<div>\r\n	There is a significant abscond rate of 25.1% among the patients studied. This high percentage influences significant early revisit to the ED. Factors contributing to this include early termination of nebulizer therapy and no discharge medicine among patients who left abruptly. This was due to a substantial number of patients who did not complete asthma treatment in ED and absconded early during subsequent visits. This problem is not unique to this hospital alone and should apply to other Kementerian Kesihatan Malaysia (KKM) hospitals as well although their percentage may vary. In contrast, this situation was not observed at Universiti Kebangsaan Malaysia Medical Centre (UKMMC) due to dedicated staff for asthma cases and hence shorter waiting time. Higher ED registration fees at UKMMC compared to KKM is also a contributing factor.</div>\r\n<div>\r\n	Concurrent infection is a contributing factor to early ED revisit following asthma treatment. There is a significant correlation between concurrent infection and ED revisit. This finding is of low clinical value as most of our patients presented with concurrent infection (76.7%). This factor can be subjective as it depends on the patients&rsquo; reported symptoms and clinical findings that suggest an infection such as fever, injected throat or chest x-ray features. Emerman and Cydulka (1995) found no association between patient&rsquo;s white blood cell (WBC) count and eosinophil count with relapse.</div>\r\n<div>\r\n	Oral corticosteroid prescriptions were only 24.9%, which is significantly lower than other study reports. Emerman et al. (1999) and Rowe et al. (2008) reported oral steroid prescriptions of 63% and 70.8%, respectively. Even when excluding those absconded cases, our sample still showed a prescription rate of only 33.2%. GINA report 2015 recommended oral corticosteroids to be prescribed in severe acute exarcerbation of asthma discharged home. This is because various studies had confirmed the benefit of oral corticosteroid during acute exacerbations as well as preventing relapse (Rowe et al. 2008). Not only our prescription rate was low, we also found a number of patients who were not given steroid prescription even during second revisit. This was probably due to failure to identify this group of patients who were actually revisit cases. In our study, we found no apparent association between oral steroid prescription and risk of ED revisit. This is due to the limitation of a observational study where the attending doctor were more likely to give a steroid prescription to patients with more complex conditions and higher risk of relapse, an excess that may not be completely reversed by the effects of the prescribed steroid.</div>\r\n<div>\r\n	Many clinical parameters that describe the severity of asthma exacerbations, including heart rate, baseline SpO2 and the number of nebulizations needed were not shown to predict ED revisit. This is consistent with studies done by Emerman et al. (1999) and Rowe et al. (2008). Additionally, they also found that peak expiratory flow rate (PEFR) does not predict relapse outcome. The present study did not record patients who relapsed and attended other hospitals or clinics. Therefore, it is possible that healthcare revisit in acute asthma exacerbation could be higher than 13.9%. This study is also a retrospective chart review, hence there will be significant biases and observation depends heavily on accurate record keeping. It also did not mention the severity of asthma according to PEFR because of inadequate data.</div>\r\n<div>\r\n	Further studies should explore the reasons for patients to abscond from a complete treatment of AEBA at the ED. Corrective measures can then be outlined and later implemented to see if it is effective in reducing asthma revisits. This can be done as a quality assurance project in the department.</div>\r\n<div>\r\n	Efforts should be made to identify patients who frequently revisit the ED for AEBA. This can be done by history taking alone by the attending doctor or assistant medical officer. Alternatively, a computerized system can be designed to reveal the number of recent ED visits when a particular patient registers at the registration counter. Identifying this group of patients will greatly help in the management as we can look into their compliance, inhaler techniques, deciding on longer observation in the ED or even admission. Furthermore, the attending doctor can refer this group of patients to asthma educators, pharmacists or respiratory clinic for further management in the area of asthma control.</div>\r\n<div>\r\n	More emphasis should be given on the prescription of oral corticosteroid in AEBA patients discharged from the ED. This can be carried out as part of regular CME programs in the department. A simple guideline or standard operating procedure (SOP) can also be made to facilitate new medical officers in attending asthma cases.</div>\r\n<div>\r\n	In addition to this a more efficient system for attending patients who received nebulizers in ED to avoid high level of abscond patients. Patients who went home before being assessed have a higher risk for ED revisit. This reflects to inadequate treatment and increase mortality and morbidity. Doctors should be informed once the patients has finished their first nebulizers and not let patients waiting for a long time.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The revisit rate of AEBA patients discharged from the ED remained high. This is partly due to high abscond rate and perhaps low prescription rate of oral corticosteroid. Concurrent infection was also shown to be associated with early revisit. These data can be useful for clinicians to identify patients at risk and for the development of ED guidelines, educational strategies and follow-up plans for the prevention of asthma relapses.</div>\r\n', 1),
(7273, 7264, '<p>\r\n	A basic principle of hospitalization is to provide adequate definitive care and treatment. In the past, medical &lsquo;paternalism&rsquo; meant that patients were unable to make their own decisions regarding nature of their medical care (Bell et al. 1847). However, patients are now more aware of their rights to decide and determine these aspects. The expression of these rights of self-determination may result in a patient discharging themselves against medical advice. Discharge against medical&nbsp;advice (DAMA) refers to a patient who leaves hospital before the treating physician recommends discharge (Alfandre 2009). Premature withdrawal from treatment has been shown to result in higher rates of hospital readmissions (Hwang et al. 2003). These patients are potentially susceptible to life-threatening consequences in the absence of optimal medical care (Carrese 2006; Moy &amp; Bartman 1996). Readmission has been shown to lead to longer hospital stays, higher costs of care, increased use of hospital&nbsp;<span style="font-size: 12px; line-height: normal;">delivery and quality of care to non-DAMA patients (Moy &amp; Bartman 1996; Ibrahim et al. 2007). This results in an increase in burden of health care facilities. The present study aimed to determine the factors associated with DAMA in an Emergency Department (ED) in order to better understand and minimize DAMA.</span></p>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a prospective unmatched case control study conducted over a 4-month period. Data was collected following the approval of the institutional review board and ethics committee. The cases were patients who took DAMA; controls were patients who were admitted to an in-patient ward from the ED. For every DAMA, two control patients were randomly selected on the same day. Using a Power Sample Size Calculator program, a total of 26 cases of patients and 52 controls were required to achieve statistical significance of 0.05 and power of 80 percent. Calculation was made based on the proportions quoted in the study by Baptist et al. (2007).</div>\r\n<div>\r\n	The exclusion criteria were patients who were less than eighteen years of age, non-resident, transferred patients, incomplete physician&rsquo;s evaluation, or with reduced Glasgow Coma Scale (GCS), intubated patients and those who were not contactable within two weeks. Data was collected following patient&rsquo;s consent. A standardized questionnaire was used to elicit factors that influenced the personal and treatment factors affecting their decision-making pertaining to the DAMA episode. Patients were contacted by the investigator for information regarding hospitalization within two weeks of DAMA. Statistical Package for Social Sciences (SPSS) version 19 was used to analyse the data. Univariate analysis was performed to identify significant factors. Important influencing factors and factors with significance of less than 0.2 were included in the final multiple logistic regression analysis.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 93 patients had given their consent for the study. There were 31 patients in the DAMA group and 62 patients in the control group however, two patients in the control group did not complete the questionnaire form as indicated, thus were not included in the analysis for factors influencing DAMA. There appeared to be no statistical demographic differences between the control and DAMA groups (Table 1). The method of hospital payment shows a significant association in the decision-making process. Self-paying patients were three times more likely to DAMA (Table 2). Within two weeks after DAMA, a total of 80.6% patients were readmitted to hospital. Patients citing family and work obligations were four times more likely to DAMA (Table 3). Expectation of an &lsquo;immediate&rsquo; cure of the disease significantly influenced DAMA (p=0.02). Multiple logistic regression analysis identified that self-paying patients were more likely to DAMA than those who had a guarantee letter (GL) or insurance cover (Table 4). The principal reasons given for DAMA&nbsp;were achievement of relief of symptoms (19.4%), financial problems (16.1%), living far away from the hospital (16.1%), dissatisfaction with care (12.9%) and work obligations (9.7%). DISCUSSION</div>\r\n<div>\r\n	It is important for doctors in the Emergency Department to identify potential DAMA patients early so that intervention can be undertaken to prevent DAMA. In previous studies, DAMA patients were found to be young males from low socio-economic status groups and without health insurance (Ibrahim et al. 2007; Nasir &amp; Babalola 2008; Pennycook et al. 1992). The present study found a significant association of DAMA with family and work obligations but not in childcare. Traditionally, the main family earners are males. Hence, their absence from work will affect the family&rsquo;s income (Baptist et al. 2007; Onukwugha et al. 2010). The majority of our DAMA patients were from lower-income groups. One third attributed financial difficulties as the major factor for their decision to DAMA. Self-paying patients were three times more likely to take DAMA. This finding is comparable to previous studies (Ibrahim et al. 2007; Nasir &amp; Babalola 2008; Fiscella et al. 2007).This factor is potentially open to modification as policies may be adopted to ease the burden of patients with financial difficulties. Free registration, discounted charges, payment planning scheme, access to social welfare support and alliances with non-governmental organizations or corporate bodies should be considered.</div>\r\n<div>\r\n	Higher rates of DAMA were found in older age groups, patients with HIV infection, alcohol intoxication, substance abuse and psychiatric conditions (Green et al. 2004). DAMA in older age groups is especially of concern as these patients may subsequently experience more complications as they may have higher prevalence of co-morbidities. Furthermore, these patients may return with increased disease severity resulting in prolonged hospital stay, and higher rates of morbidity and mortality (Green et al. 2004).</div>\r\n<div>\r\n	Increased DAMA was identified in patients with lower educational backgrounds. This is most likely due to poor understanding of their disease and treatment requirements. Health care providers may have used medical terms during explanation to patients regarding the nature of the disease, its course and compliance to treatment. Having good literacy is important to access the health care system. Patients with limited literacy will have increased reliance on health care personnel for assistance and advice and this will affect health outcomes (Osborn et al. 2011; Morris et al. 2011).</div>\r\n<div>\r\n	Good documentation practice should include information regarding the disease and its severity, risk and benefit of hospitalization and treatment, patient&rsquo;s decision-making competency and arrangements for follow-up or an alternative care (Devitt et al. 2000a; Devitt et al. 2000b).There are multiple tools available to assess patient&rsquo;s decision-making capacity but they are time-consuming for use in the ED (Grisso &amp; Appelbaum 1998; Appelbaum 2007; Kim &amp; Caine 2002).Any assessment for competency in decision-making for DAMA is based on four steps by the patient: i) ability to understand the information provided; ii) ability to retain this information; iii) ability to process the information; iv) ability to communicate the decision clearly (Phillips et al. 2009). A decision to prematurely withdraw from treatment has an adverse health effects resulting in higher rates of ED revisits and hospital readmissions (Hwang et al. 2003; Fiscella et al. 2007). These increase the burden on healthcare facilities as readmissions result in longer hospital stays, higher costs of care, increased use of hospital resources, and indirectly affects the delivery and quality of care to non-DAMA patients (Moy &amp; Bartman 1996; Ibrahim et al. 2007; Seaborn Moyse &amp; Osmun 2004). Interventions such as following up DAMA patients by phone calls, home visits and arrangement of proper outpatient care is recommended (Hwang et al. 2003).</div>\r\n<div>\r\n	Quality of care is the fundamental basis of a health care institution. Communication plays a major role in the provision of quality medical care. Inconsistency of information, unavailability of healthcare providers for enquiry or explanation, apathy, rudeness and unfriendly welcome have previously been shown to be contributing factors for DAMA (Pennycook et al. 1992; Onukwugha et al. 2010; Berger 2008).</div>\r\n<div>\r\n	The results of the present study found that the tendency to DAMA was higher in patients who experienced symptomatic relief of their condition. Immediate relief of symptoms can result in a belief of cure (Wong et al. 2000). Trust is a fundamental component in the clinician-patient relationship. Confidence in a physician&rsquo;s care increases the patient compliance and adherence to treatment; without this patients are more likely to seek a second opinion (Nasir &amp; Babalola 2008; Mckinstry et al. 2006). Cultural beliefs and alternative care seeking behaviour from traditional healers may influence our society and in other studies, it was found to contribute to DAMA (Nasir &amp; Babalola 2008). The majority of patients agreed to the prescribed treatment plan, but a few disagreed due to a strong belief in traditional complimentary medicine. Inconsistency of a treatment plan from one physician to another, patients of young age, higher educational background, increased self-determination and the capability of making decisions have been shown to contribute to DAMA (Onukwugha et al. 2010; Wong et al. 2000). Taking adequate time to explain about the nature of the patient&rsquo;s condition and its course may generate the trust and gain confidence from the patient, therefore preventing DAMA.</div>\r\n<div>\r\n	Every physician is obligated to protect their patients from harm and to promote well-being. However, patients can practice their autonomy to choose or refuse a course of treatment (Carrese 2006). Striking a balance between these two fundamental, but potentially conflicting, obligations is a challenge. The present study confirms that readmission rates are high in DAMA patients, implying that they have left prematurely and with suboptimal treatment. To address this, we suggest that a standard DAMA protocol must be followed. This includes providing choice of treatment, informing the patient about likely sequelae of DAMA, determining their competency in decision-making and instituting appropriate follow-up after a DAMA episode (Berger 2008; Naess et al. 2001). This study was limited to a single centre which may not reflect the DAMA situation for other institution in this country.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Financial issues have a significant impact on DAMA. By identifying factors which influences patient&rsquo;s decisions to take DAMA, early appropriate measures and effective preventive strategies may be undertaken to minimize DAMA. ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors are grateful to Professor Colin Robertson (University of Edinburgh) for reviewing this manuscript. We would like to thank Mrs Nurul Saadah Ahmad and Mrs Saidah Mohd Hasbi (Centre of Research in Emergency Medicine CREM) for technical support.This research was funded by Universiti Kebangsaan Malaysia Medical Centre (UKMMC) Fundamental Research Fund [FF-276-2011].</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(7280, 7271, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Cardiac arrest is a medical emergency. It is defined as the cessation of normal blood circulation due to failure of the heart to contract effectively to produce sufficient cardiac output. At least one third of the world population is at high risk of developing major cardiovascular events due to tobacco use, overweight, elevated blood pressure, blood cholesterol and diabetes (World Health Organization 2002). An estimated 17 million people die of cardiovascular disease annually (World Health Organization 2002). The majority of these deaths occur in out of the hospital setting. All out-of-hospital cardiac arrest (OHCA) patients require immediate cardiopulmonary resuscitation (CPR) and emergency medical intervention irrespective of the underlying cause. For every minute of delay in initiating CPR, the chance of survival decreases (American Heart Association in collaboration with International Liaison Committee on Resuscitation 2000; Travers et al. 2010; Berg et al. 2010). According to the American Heart Association 2010 guideline, CPR and defibrillation within 4 mins and the commencement of paramedic helpwithin 8 minutes of collapsed increases the chance of surviving sudden cardiac arrest to 43%. This emphasizes the importance of early initiation of CPR before the arrival of paramedics or before arriving at the emergency department (ED). To ensure each link in the survival chain works successfully, more initiatives are needed to increase public awareness about the importance of initiating early CPR. The guidelines also emphasize on the importance of chest compression without mouth-to-mouth ventilation (Travers et al. 2010; Berg et al. 2010). Chest compression without mouth-to-mouth ventilation is perhaps more acceptable to the public and easier to perform and teach. Studies on the prevalence of bystander CPR in Kota Bharu community in Kelantan, Malaysia, showed poor public response to initiate early CPR following OHCA and poor outcome (Hisamuddin et al. 2007; Chew et al. 2008a). More efforts are needed to increase the community awareness in Malaysia about the importance of initiating early CPR.</div>\r\n<div>\r\n	Kuala Lumpur is a Federal Territory and the capital of Malaysia. Both private and government ambulance organisations serve the community especially in urban areas like Kuala Lumpur. Government ambulance services are provided by Ministry of Health, Ministry of Education and Civil Defence. Private ambulance services are run by non-governmental organization such as Red Crescent and St. John&rsquo;s Ambulance. Literature search did not reveal any study related to the incidence of bystander CPR in OHCA in Kuala Lumpur.</div>\r\n<div>\r\n	The aim of the present study was to determine the prevalence of bystander OHCA CPR and to identify the baseline survival rate of OHCA patients managed in the ED of Universiti Kebangsaan Malaysia Medical Centre (UKMMC).</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a cross-sectional study of OHCA patients resuscitated in the ED of Universiti Kebangsaan Malaysia Medical Centre from January to December 2010. Data was collected following the approval of the institutional review board and ethics committee. The medical registration numbers of OHCA patients were obtained from the resuscitation logbook and respective patient&rsquo;s files were reviewed. All information was documented in a standardized data collection sheet. Only patients who arrested out of the hospital and managed in the ED were analysed. Early CPR is defined as CPR initiated following cardiac arrest prior to the arrival of paramedics or arrival to the ED. Data collected included: patients&rsquo; age, gender, race, chief complaint and past medical history. The variables recorded were: aetiology of arrest, initiation of CPR at the scene, the application of automated external defibrillator (AED), mode of transportation and the incidence of ROSC. The time of collapse, CPR initiation, ED arrival, ROSC and CPR ended were documented. Descriptive data analysis was used to analyse the demographic characteristics of the patients. Categorical data was analysed using chi-square test and fisher exact test (p-value &lt; 0.05). Data was analysed using Statistical Package for the Social Sciences version 19.0 (SPSS, Inc., Chicago, IL). &nbsp;RESULTS</div>\r\n<div>\r\n	A total of 117 OHCA patients were recruited at ED, UKMMC within the study period. Ninety-eight patients fulfilled the study criteria for final analysis. Nineteen patients had incomplete data. The mean age for OHCA patients was 48 years (range 1 month - 83 years) and 75% of the patients were males. The majority ethnic group was Chinese (45%) followed by Malays (34%), Indians (14%) and others (7%).</div>\r\n<div>\r\n	Three patients received early CPR (Table 1). Patients who received early CPR achieved higher rate of ROSC (33%) compared to those who received late CPR (20%). A higher rate of ROSC was found in female OHCA patients (39%) compared to the male counterparts (15%). The majority (64%) of OHCA were witnessed. PHC personnel witnessed 6% of OHCA and the rest was witnessed by family member or other bystanders. Eleven OHCA patients received bystander CPR. Forty-seven (48%) of OHCA patients had CPR initiated at the scene. PHC personnel initiated CPR in 36 OHCA patients and achieved higher percentage of ROSC (25%). Nine trauma related OHCA showed a higher rate of ROSC (33%) compared to 89 non-trauma related arrest with 21% ROSC. However, this association was not statistically significant. Automated external defibrillators were applied to 28 patients. Of these, two had shock able rhythm with shock delivered but neither achieved ROSC. Only PHC personnel used AED. Seventy-three patients had a significant past medical history (PMH) such as hypertension, diabetes mellitus and ischaemic heart disease. There was no significant difference in the presence of PMH between male and female patients. Forty percent of female patients achieved ROSC compared to 16% in male patients. Pre-hospital care ambulance services transported 43% of the patients to the ED. The remainder were brought to the ED using other modes of transportation. Higher percentages of ROSC (23%) were achieved in those that were brought by other modes compared to those brought in by PHC services. The mean (standard deviation) duration from collapse to ED arrival was 41 (27) mins in patients transported by PHC services and 29 (24) mins by other modes of transportation (Table 2). Twenty-two of the 98 OHCA patients achieved ROSC after receiving further resuscitation in ED. Of the 22 ROSC patients, nine (41%) survived to ward admission for definitive care. The mean (standard deviation) duration of survival was 49 (34) hours. The longest survival duration in the ward was 92.5 hrs.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The UKMMC serves the community of Cheras, in the southern region of the Federal Territory of Kuala Lumpur. This study represents a first look at the outcome of bystander initiated CPR on OHCA patients from this area. The percentage of bystander CPR in the Cheras community (11%) was higher than in the Kota Bharu community (9%) (Hisamuddin et al. 2007). However, these figures are still poor compared to the worldwide average level of 33% (ECC Committee, Subcommittees and Task Forces of the American Heart Association 2005). These marked differences indirectly reflect the level of awareness and willingness of Cheras and Kota Bharu communities to perform CPR in OHCA. The public should be made aware that approximately 70% of OHCA occurs at home involving family members (Weaver et al. 1984). Early bystander initiated CPR can double the probability of survival of these patients (Finn et al. 2001). Early CPR improves survival by delaying the degradation of the ventricular fibrillation or ventricular tachycardia to a systole and enhances the possibility of successful defibrillation on arrival of AED-equipped paramedics (Finn et al. 2001). Bystander initiated CPR &lsquo;buys time&rsquo; during this time-critical situation. Therefore, by increasing the incidence of bystander CPR in a community should also increase the overall survival of OHCA.</div>\r\n<div>\r\n	Of the 65-witnessed arrests documented in this study, only nine received out-of-hospital CPR. Three OHCA CPR were categorized as early and all were initiated by healthcare providers. Although not statistically significant, OHCA patients who received early CPR appear to have a better outcome compared to those receiving late CPR. It was identified from the documented history that CPR performed by bystanders had indeterminate periods of discontinuity. Several CPR were performed for a short duration prior to transportation without resuming CPR in their vehicle. These inadequacies may have influenced the outcome of OHCA. Early CPR is a vital link in the chain of survival (ECC Committee, Subcommittees and Task Forces of the American Heart Association 2005). To maximize success, bystander CPR should be applied within 4 mins of collapse. The quality and technique of CPR performed as well as the time interval between collapse and the start of bystander CPR are equally important determinants for OHCA outcome (Hisamuddin et al. 2007).</div>\r\n<div>\r\n	In our study, female OHCA patients appear to have a higher percentage of ROSC compare to male patients (p=0.015). This finding is comparable to the study in Sweden, which reported that female OHCA was a predictor of higher rate of survival (Herlitz et al. 2004). This is explained by the pre-morbid medical conditions in both sexes. Male patients in our study had a higher incidence of severe pre-morbid medical conditions such as ischemic heart disease and hypertension compared to female patients even though the incidence of PMH in both sexes was not statistically significant. Patient&rsquo;s gender may influence the outcome of OHCA (Herlitz et al. 2004; Andjelic &amp; Djordjevic 2010). The more severe the pre-morbid conditions or medical history of a patient, the poorer is the outcome.</div>\r\n<div>\r\n	In our study, PHC personnel initiated most of the CPR performed at the scene. The OHCA patients that received PHC CPR have achieved more ROSC in the ED compared to bystander CPR. However, there was no difference in the survival outcome in both groups. There were only two OHCA patients recognized by AED as having shockable rhythm. PHC personnel delivered shock to these two patients but they did not achieve ROSC. Most of the initial cardiac arrest rhythms identified in this study were non-shockable rhythms, which was asystole. These findings are comparable to the study of OHCA patients in Hong Kong (Lau et al. 2005). A study of the emergency medical services system in Chicago, which served more than 3 million inhabitants, showed approximately 10% of OHCA occurred after the arrival of emergency personnel (Becker et al. 1991). The trend of outcomes of OHCA witnessed by PHC personnel was not statistically significant compared to bystander-witnessed arrest. It is believed that besides providing good quality CPR, PHC personnel have the additional advantage of having an AED in the ambulance. Moreno Martin et al. (2009) demonstrated the effectiveness of having AEDs in all BLS ambulance units. Defibrillation for shockable rhythms can be delivered before patient&rsquo;s arrival to the ED. Survival with complete recovery was observed in 7% of OHCA patients. This figure is similar to the results for recovery in ED (12%), which included the arrests attended directly by ACLS equipped and trained services (Moreno Martin et al. 2009).This shows the advantage of being equipped with this device at all times. The AED was found to be safe even in the hands of untrained schoolchildren (Gundry et al. 1999). By placing AED in strategic public locations, the time from collapse to application of the first shock was significantly shortened (Valenzuela et al. 2000; Israel &amp; Hohnloser 2006).</div>\r\n<div>\r\n	Higher percentages of ROSC were noted in OHCA patients transported to the ED by other modes of transport. These OHCA patients had collapsed at home and were witnessed by family members. The favourable outcomes were probably due to shorter time intervals from time of collapsed to the initiation of ACLS in the ED.</div>\r\n<div>\r\n	It appears that trauma related OHCA in our study have better outcome compared to non-trauma. Our result seems to differ from the study by Chew et al. (2008b) that suggested the percentage of non-trauma related cases have a better outcome in terms of achieving ROSC. Very low survival rates have been reported for victims of traumatic cardiac arrest (Stockinger &amp; McSwain 2004). CPR might be useful in severe trauma patients only in systems with very short transportation times (Pepe &amp; Eckstein 1998). Those patients with the best outcome from trauma arrest generally are young, have treatable penetrating injuries, have received out-of-hospital endotracheal intubation, and prompt transport (less than ten minutes) to a trauma care facility (Chew et al. 2008b; Pepe &amp; Eckstein 1998).</div>\r\n<div>\r\n	There are several limitations in this study. The sample size was small. There were only 98 incidence included for analysis over a limited period of 12 months. This study did not analyse the quality, skills and knowledge of bystanders performing CPR. Further study is needed to determine the association of ED management with the outcome of resuscitating OHCA patients.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The provision of early CPR for OHCA by the public in the Cheras area is low and AED was not widely available for public use. Females have a better rate of ROSC than males. To improve outcomes, more effort is required to increase the awareness and involvement of the public in managing OHCA. ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to thank Professor Colin Robertson from University of Edinburgh, for reviewing this manuscript. We thank Dr. Hushairy Harunarashid, Dr. Zuraidah Che Man, Ms Saadah Ahmad and Ms Saidah Mohd Hasbi from Centre of Research in Emergency Medicine (CREM) of Universiti Kebangsaan Malaysia Medical Centre (UKMMC) for the technical support. The funding for this research was provided by the UKMMC Fundamental Research Fund [FF-135-2011].</div>\r\n', 1),
(7283, 7274, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	The use of multimedia instruction like video, has been established as a potential teaching technique. The Video assisted learning method is similar to traditional lectures. In few cases, it increased student learning satisfaction (Lee et al. 2007). A study by Akl et al. (2008) supported the capability of students&rsquo; performance which increased with video assisted learning. In the same study, the authors found that the students were able to complete laparoscopic suturing procedure within an accepted time. In another study by Xeroulis et al. (2007), sixty first year medical students were taught on suturing with the use of self study computer-based video instructionand two blinded experts assessed their performance on a Global Rating Scale which has shown improvement in students&rsquo; suturing skills. In addition, study by Lee et al. (2007) also concluded that the use of an instructional DVD is more effective&nbsp;than face-to-face didactic teaching for teaching pediatric intra-osseous needle insertion to medical professionals. Furthermore, a study by Whatley and Ahmad (2007) which assessed the use of video as a teaching tool in medical education showed that video is a powerful teaching medium to attract students&rsquo; attention. The study also revealed that it is also a strong motivator for learning. Moreover, over the years, videos have gained popularity on the web as a medium of presenting materials that incorporate multimedia content for e-learning. This method allows learners to learn on their own using video. However, according to Hammound et al. (2006) it is difficult to find and develop effective and quality video-module as an instructional material. Currently, there are a large number of poorly designed instructional videos frequently accessible through different venues such as the internet or commercially available videos that provide very limited instruction. Valcke and Wever (2006) has mentioned, that the current concern is to find adequate information to develop effective and appealingly designed video instruction materials. Nevertheless, a qualitative perception of using video as a learning medium needs to be carried out to explore further on how video based learning can be optimized in teaching healthcare providers.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This study used a qualitative research design. This study aimed at exploring paramedic students&rsquo; perception of the video assisted learning method as an instructional method of learning emergency skills.</div>\r\n<div>\r\n	Ethical approval was obtained from the Medical Research Ethics Committee, Universiti Kebangsaan Malaysia Medical Centre prior to data collection. During the whole research process thought was given to non-compulsion, educated assent, members&rsquo; secrecy, flexibility of members to pull back at whatever time they wished without being penalized. The members were consoled that their reactions would be kept secret and their characters would not be uncovered in research report and publications. They were recognized by the use of pseudonyms.</div>\r\n<div>\r\n	PARTICIPANTS</div>\r\n<div>\r\n	Twenty paramedics were involved in this study. They attended a 15- minute video session on knowledge and skills regarding intraosseous cannulation in English. Then they were assigned into four groups and each individual in the group practiced the skills. After the practice session, they were assessed on their knowledge and skills. After completing the session, all the paramedics were invited to be involved in a focus group discussion pertaining to their experiences of the teaching method. All of them participated in this study. They were divided into four groups and each group consisted of five participants.</div>\r\n<div>\r\n	DATA COLLECTION</div>\r\n<div>\r\n	A semi&ndash;structured discussion guide was used to collect data on the participants&rsquo; insight regarding the use of video for enhancement of knowledge and psychomotor skills. The interview was designed to explore their perception regarding video assisted learning. The data were collected through focus group discussion. The discussion was conducted after the training session. The session started with a briefing to all the participants regarding the purpose and method involved in the discussion. A total of four group discussion sessions were recorded with sound recording record-keeper after getting informed consent from them. Each focus group discussion lasted for about 45 minutes. Three general questions were discussed &ldquo;how do you feel about this teaching session&rdquo;, &ldquo;what is your expectation of using video as a teaching tool&rdquo; and &ldquo;how do you suggest to improve the delivery of teaching by video&rdquo;. Various methods of probing such as active listening, paraphrasing, and reflecting were employed to facilitate the discussion. DATA ANALYSIS</div>\r\n<div>\r\n	The qualitative data was collected and analyzed, concurrently. A content analysis method was performed whereby an orderly coding and sorting methodology was utilized. The researcher listened to the audio recorded talk painstakingly and information was translated verbatim. Transcribed information was analyzed through the constant comparative; a back and forth process to identify the themes which were coded and categorized.</div>\r\n<div>\r\n	Concerns of validity and reliability in qualitative studies such as this concerns its trustworthiness. The first measure of trustworthiness was gathered through member-checking where three of the participants checked and affirmed the themes identified. Then, two experts from the medical staff were given the data and asked to identify the themes and develop the codes and categories. These were compared to those identified and developed by the researcher.</div>\r\n<div>\r\n	RESULTSDEMOGRAPHICS CHARACTERISTICS OF PARTICIPANTS</div>\r\n<div>\r\n	The sample consisted of 20 paramedics. The study had a total of 2 females and 18 males. Their ages were between 21-32 years. Fifty-five percent of the participants had more than 3 years of working experience and 45 % had less than 3 years of working experiences. All the participants were diploma holders of a Diploma in Medical Assistance Programme.</div>\r\n<div>\r\n	EMERGING THEMES: PERCEPTIONS TOWARD VIDEO ASSISTED LEARNING METHODOLOGY ON LEARNING EMERGENCY SKILLS</div>\r\n<div>\r\n	This section presents the findings from the discussion, according to the themes identified through the qualitative data analysis process. Three major themes were identified which were categorized as: i) advantages of video as a teaching tool, ii) barrier in using video as a teaching tool; and iii) suggestions on using video as a teaching tool.</div>\r\n<div>\r\n	ADVANTAGES OF VIDEO AS TEACHING TOOL</div>\r\n<div>\r\n	Eight sub-themes were identified from the discussions which were categorized as advantages of video as a teaching tool. The participants felt that the use of video in the training was an advantage because it: (i) increased their interest; (ii) provided better understanding; (iii) presented a better learning tool; (iv) increased their confidence; (v) promoted self-learning; (vi) enabled observation of learning by doing; (vii) helped them in learning procedure skills; and (viii) helped them in delivering standard information.</div>\r\n<div>\r\n	When asked about how the video was a teaching tool, participants said that they were happy using the video and felt that it increased their interest because [they could] see how the skill was done by watching and it [made] clearer picture of the procedure, really see what [was] happening &hellip; language that they used in teaching is simple and easy to understand and that the video has an image and action with a voice which we can watch than lecture. In terms of understanding, the participants felt that the video provided better understanding because they could see the steps one-by-one with an explanation which [gave] better understanding &hellip; learn better &hellip; see the way how a skill was demonstrated clearly on the picture and [could] focus to it, and the image of the equipment and place and procedure. However, one of the participants - mentioned that it was a new method and he needed someone to show the steps to him. He felt that he could not fully learn from the video; that he could only get 60-70% of information from this video because [he felt] difficulty in understanding the language and [he was] not very good in English. He preferred that the medium of instruction to be his mother tongue which is Malay to make him understand better.</div>\r\n<div>\r\n	In addition, one of the participants also felt that when [he] focused [he could] learn the steps by watching the video [and] concentrated [his] view and mind to the video&hellip; because [he was] interested with the skill. It [made his] mind capture a picture of the video. Meanwhile, one of female participants felt that [she could] see many times if it is in the video. [They could] play on DVD and watch as many times [they wanted]. By doing so, [they] understand and remember better. [They] also [could] pause [the] video and watch properly, the video to look at all the steps. However, they felt not able to catch the word &hellip; very fast &hellip; [could] understand but not fully.</div>\r\n<div>\r\n	The participants also felt that the use of instructional video in learning intraosseous cannulation made them more confident in performing the skill. They felt by watching the video then practice could improve their confidence level. They felt that [they could] see many times and replay and replay to remember the steps &hellip; to practice while watching the video &hellip; gain more confident when practice together with video ; that the video was very helpful [because they could] perform more confidently when [they could] see the skills. It also [helped them] to learn any time when free.</div>\r\n<div>\r\n	In terms of promoting self-directed learning, only a few participants responded clearly that the video promoted self-directed learning. He said that there are many emergency skills to be learned and it is difficult to remember all of them. So, [he preferred] to watch video to enhance [his] skills. This is because not all the skills [are] practiced every day like Cardiopulmonary Resuscitation (CPR), which [they could] practice and remember better. Meanwhile, although others did not respond directly that the video promoted self-directed learning, one participant responded that [she] sometimes watch the video for enhancing [her] skill from YouTube indicates that she was comparing the instructional video she used in this study was like the video that she observed on You Tube which required her to access on her own. Similarly with, similar to others&rsquo; response that they could use video to recall the skills indicate that the video did promote self-directed learning.</div>\r\n<div>\r\n	Other advantage of the use of the instructional video was that it enabled observation of learning by doing, the learning of procedural skills and delivering of standard information. Learning occurs more when the input is not only through listening or audio, but also through watching or visual. This was specifically indicated by a participant who said that [he] learned the skill by watching from another person doing and sometimes [teaching] by senior. This implies that the video enabled him to observe another person doing the action and therefore learn from that observation. The participants also mentioned that video can help in learning of procedural skill because they could use video to recall the procedure steps, and it enabled standard information to be conveyed accurately. This was because a recorded video could be used to replay whenever necessary.</div>\r\n<div>\r\n	BARRIER IN USING VIDEO AS A TEACHING TOOL</div>\r\n<div>\r\n	Result from the discussion showed that there was only one barrier in learning through video. The participants said that they had a problem in learning through video because of the medium of instruction which was difficult to understand. They preferred the language to be Malay which they felt necessary to enhance their acquisition and learning of knowledge and psychomotor skills. They said that [it would be] helpful, but need to focus on the language and pronunciation which [they] need time to interpret and understand it and they [had] difficulty in following different language very fast. Other than Malay language displayed video, [they] needed more time to watch the video before [they could perform the procedure). If the video was in Malay language (they) had no problem to watch and follow the video contents.</div>\r\n<div>\r\n	SUGGESTIONS IN USING VIDEO AS A TEACHING TOOL</div>\r\n<div>\r\n	Based on the barrier described by participants, it appeared that the only problem was the language used in the video. Therefore, they suggested that the language of instruction should be in Malay so as to enhance their understanding which may contribute to their acquisition and retention of the knowledge and skills. If the language used in the video was Malay, [it was] easier to understand the steps better because [the] language was mother tongue language (Ms X). One of the participants (she) also felt that apart from watching the video, she [needed] someone to teach her. She expressed her worries because [she is] still a junior staff and probably [make] mistake if [she practiced by herself] without a guide. She said that she was still learning and it [was risky] for [her] to practice [by herself]. Therefore, (she) still needed trainer to teach the emergency skill, although we watched the video &hellip; She also felt that she needed to read and understand before watching the video and it can &hellip; make to understand better.</div>\r\n<div>\r\n	In addition, the participants felt that video could be used to learn some short emergency skills. This is because they could recall the skills when they watched the video. They felt that it was easier to remember and could help in doing revision. DISCUSSION</div>\r\n<div>\r\n	In this study, the analysis of perception of the four focus groups&rsquo; discussion towards VAL showed that the participants felt video had a positive learning impact regarding learning emergency skills. They expressed their feeling on the use of video as a teaching tool for emergency skills. They felt that the video plays an important role in the teaching of emergency skill. The finding noted the advantages of video on teaching of emergency skill. This means that they felt the video was a better teaching tool for learning emergency skills. A similar study by Kenny (2002) found that participants felt the use of video for learning emergency skills was a flexible learning method. Thiele (2003) found that students were more motivated in the self-preparation learning journey. Majority of the students had dissatisfactory feelings of being unable to ask questions while watching a video. This reflects to theories that propagate the use of multimedia tools as an engaging tools for students learning.</div>\r\n<div>\r\n	Tsay et al. (2000) expressed that learning occurs when a learner constructs an internal representation of his / her unique vision of knowledge. Moreover, Leidner and Jarvenpaa (1995) found that individuals would learn better when they discover things by themselves. This is similar to the discussion findings when the participants mentioned that they like to learn through video because they felt that intraosseous cannulation is an important skill for them. In this study, the participants felt that video engaged them to learn the skill. This is because they felt that video has images and action. It was similar to the finding by Zhang et al. (2006) that video allowed students to view actual objects and realistic scenes, to see sequences in motion and to listen to the narration.</div>\r\n<div>\r\n	Despite findings indicate that video is one of the learning tools for teaching and learning of emergency skills;the participants also suggested ways to improve the video provided so as to gather more potentials of using video in learning. The participants suggested that the language of instruction should be the participants&rsquo; mother tongue in order for them to understand the content better. They also suggested that the contents of the lecture and information delivery has to be considered for attracting more attention and engaging the students. They also suggested that it is a suitable tool for learning short emergency skills which needs to be considered in preparation of the video.</div>\r\n<div>\r\n	In this study, all the four focussed groups&rsquo; discussion expressed similar perceptions towards the use of video as a teaching tool for emergency skills. Although, the paramedics differ in their age, working experience and education background, they were similar in their perception; which they felt that the video has helped them to improve their knowledge and skills on emergency procedure. Nevertheless, one paramedic said that the video did not play an important role in the teaching of emergency procedure because he felt that he needed an instructor to guide him. This feeling was illuminated in his performance where he did not perform well in both the knowledge and skills tasks after being exposed to the video. This paramedic was the most senior participant in comparison with the other paramedics. This implies that to some extent, age difference could also influence the finding. The young paramedics were more enthusiastic to learn in comparison to the senior staff; who may also be less motivated after been working for many years. Another factor that could influence their perception towards VAL was their training experience. Findings indicate that the participants&rsquo; training level could also influence their perception. This is because those who attended basic and advanced training felt the video as a helpful tool to gain information, but those with basic training only felt that video was not the only tool to teach skills. This is because; currently the education of emergency training using video has become more common. Those that have been trained in advanced courses were usually exposed to a video orientated training program rather than basic training. For instance, training of Advance Cardiac Life Support usually conducted with the use of video. By doing so, the participants exposed to this method are encouraged to do self-directed learning before attending the course. This could influence their motivation on starting self-directed learning in the future.</div>\r\n<div>\r\n	The participants also felt that the language used should be in their mother tongue. In Malaysia, Bahasa Melayu is the formal and compulsory language while English is the second language. This means that more Malaysians are better versed in the Malay language rather than English. It can be said that our paramedics are more comfortable with Malay to talk, learn and write. Therefore, the preparation of video using Malay language may need to be considered in the future.</div>\r\n<div>\r\n	This research was limited by the participants of the study. The paramedics involved were only from one teaching hospital. Hence, the findings revealed these paramedics&rsquo; insights and their experienc with VAL.</div>\r\n<div>\r\n	The analysis of qualitative data showed that participants could also suggest on the development of the video; in relation to the medium of instruction used. It needs to be in a language that is suitable and easy to understand. This suggestion can be considered in the development of future videos. The participants preferred that the language used in this video to be the mother tongue, so as to enhance their understanding.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	As a conclusion, this study indicated that paramedics perceived video assisted learning as a potential tool for learning emergency skills. However, they suggested the medium used for video instruction need to be in their mother tongue for better understanding.</div>\r\n', 1),
(7291, 7282, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Chronic rhinosinusitis with nasal polyposis (CRSwNP) is a chronic, non-neoplastic inflammatory disease of the paranasal sinuses, and mostly originates in the nasal mucosa of the middle meatus. The estimated incidence in the general population is 1-4% (Couto et al. 2008).</div>\r\n<div>\r\n	Research studies conducted on Caucasian population showed that eosinophil-predominant was the most common feature of NP and ranged between 73-92.5% of their sample size (Armengot et al. 2010; Garin et al. 2008; Snidvongs et al. 2012; Couto et al. 2008). However, research studies conducted in India and Asian countries showed that about 48-69% of NP was neutrophil predominant and had lower cases of eosinophil-predominant NP compared to the Caucasian studies (Dafale et al. 2012; Hao et al. 2006; Ba et al. 2011). A study showed 17% of the sample were in combination of eosinophil and neutrophil, and 27% of NP had high level of mast cell, suggesting that the pathogenic mechanism of NP involved a complex inflammation included other inflammatory cells (Hao et al. 2006). A study about the comparison of histological aspects nasal polyps among Chinese, Caucasians and Africans patients from different geographical and ethnological background showed that African patients had more severe types of NP (92% are stage 3 nasal polyps) compared to others and the numbers of eosinophilic infiltration in the African patients was the highest among all. However, there was no major histological difference in the NP obtained from African, Chinese and Caucasian patients and they suggested that nasal polyposis could be the same disease entity in this three groups studied (Lacroix et al. 2002). The aim of the present study was to view the NP specimen and to identify the phenotype of NP among Malaysians.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a retrospective study done on patients who were diagnosed with nasal polyps (NP) from January till August 2013 in Universiti Kebangsaan Malaysia Medical Centre (UKMMC). NP tissues were obtained from 122 CRSwNP patients during routine endonasal sinus surgery, at Department of Otorhinolaryngology, UKMMC. The diagnosis of NP was based on each patient&rsquo;s medical history and on the results of nasal endoscopy and computed tomography (CT).</div>\r\n<div>\r\n	All patients diagnosed with bilateral NP were included in this study. The ethic and research committee board approved the study.</div>\r\n<div>\r\n	Endoscopic physical findings were scored. The degree of nasal polyps was classified in relation to fixed anatomical landmarks in four steps: 0 = &lsquo;&lsquo;no polyposis&rsquo;&rsquo;, 1 = &lsquo;&lsquo;polyps in middle meatus only&rsquo;&rsquo;, 2 = &lsquo;&lsquo;polyps beyond middle meatus but not blocking the nose completely&rsquo;&rsquo;, 3 = &lsquo;&lsquo;polyps completely obstructing the nose&rsquo;&rsquo; (Fokkens et al. 2012).</div>\r\n<div>\r\n	Findings on CT scans were graded according to the Lund- Mackay scoring system. The mucosal abnormalities were graded as 0 (no abnormality), 1 (partial opacification), or 2 (total opacification) of the frontal, maxillary, anterior ethmoid, posterior ethmoid and sphenoid sinus, bilaterally. The ostiomeatal complexes were scored bilaterally as 0 (not occluded) or 2 (occluded). Those with CT grading more than 1 were considered positive and the maximal CT grading score is 24 (Lund &amp; Kennedy 1997).</div>\r\n<div>\r\n	The histological slides for all cases of NP were retrieved from Pathology Department of UKMMC. These samples were fixated in a formalin solution (10% formaldehyde), included in paraffin; 5&mu;m sections were made, which were hematoxylin/eosin stained. All slides were examined under light microscope with 400x magnification and a total of 10 high power fields were chosen for inflammatory cell counting. The number of neutrophils and/or eosinophils was counted, and average number of inflammatory cells for each high power field was calculated (Wen et al. 2012).</div>\r\n<div>\r\n	All statistical analyses were completed using SPSS v21.0 statistical software (SPSS, Inc, Chicago, IL). Descriptive data was presented as percentages and means +/- standard deviation (SD). Chi square analyses were used for relationships of nominal variables.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Out of total of 122 patients included in this study, 85 were males and 37 were females. The age ranged between 14-87 years (mean = 55.3 years). In the NP group, the ethnic classes were 57 Malays, 38 Chinese, and 27 Indians. Eosinophil-predominant was seen in 32.8% of patients and 67.2% were non-eosinophil predominant (Figure 1). Phenotype of NP was significantly associated with ethnicity (x&sup2; = 11.536; p &lt; 0.05) (Table 1).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The pathogenesis of CRSwNP involves complex inflammation regulated by various chemical mediators and cytokines produced by inflammatory cells in nasal mucosa (Bernstein et al. 1997). Clinically, it presents as a mass with round, smooth, translucent, soft, pale, glistening appearance, non-tender and moves with probing, and it is attached to the nasal sinus mucosa (Irfan &amp; Shamim 2009). Clinical manifestations include nasal obstruction, rhinorrhea, anosmia and/or hyposmia, headaches and general malaise (Couto et al. 2008). On histological observation of nasal polyps (NP), generally it is lined by ciliated pseudostratified epithelium, the layer itself is thickened by oedema and infiltrated with inflammatory cells, mostly eosinophils (Gar&iacute;n et al. 2008).</div>\r\n<div>\r\n	The general histopathological classification is eosinophil-predominant or neutrophil-predominant. Most histological studies of nasal polyp have been performed in Caucasian patients and data from Asian population is still lacking. The majority was found to be eosinophil-predominant, comprising 73-92.5% (Armengot et al. 2010; Garin et al. 2008; Snidvongs et al. 2012; Couto et al. 2008) whereas neutrophil-predominant was only 48-69%. A study done with 145 patients with NP in Sriraj Hospital, Thailand (Jareoncharsri et al. 2002) showed that 81.9% of NP was neutrophil-predominant, supported by study done in China (Ba et al. 2011). This suggested that Asian population might have different pathological mechanism from Caucasian population. Our study also showed that in Malaysian population, non-eosinophilic predominant polyps accounted for 67.5% thus differing from Caucasian populations.</div>\r\n<div>\r\n	The treatment modalities for eosinophilic or neutrophilic infiltration of NP differ from each other. Studies showed corticosteroid is effective to eosinophilic CRS by improving patients&rsquo; symptom scores and polyp size scores, reducing numbers of eosinophil in NP but not numbers of neutrophils or levels of their mediators (Sakuma et al. 2011; Wen et al. 2012). On the other hand, macrolides effectively reduced interleukin-8, which is an important chemo-attractant of neutrophil, and size of nasal polyps (Wen et al. 2012; Majima 2004), while it showed poor therapeutic efficacy and worse clinical response in eosinophilic NP (Haruna et al. 2009; Peric et al. 2011). By understanding the histological types of the NP, it would provide us certain prognostic information and allow specific tailored treatments.</div>\r\n<div>\r\n	There are certain limitations to consider in our study. As this study was conducted in a tertiary centre, it may not represent the entire population. Thus, a larger community based study may be necessary to validate the results. Besides, due to the retrospective nature of this study, errors such as inadequate samples and missing data were inevitable.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Non-eosinophilic predominant NPs among patients in UKMMC and this result supports the finding from other Asian countries. There was a significant association in Malaysian ethnicity with the highest percentage in Chinese population.&nbsp;</div>\r\n', 1),
(7293, 7284, NULL, NULL);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(7295, 7286, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Post-operative pain is a common problem experienced by patients following surgery. Often, it is not treated appropriately and leads to serious discomfort and delayed recovery from surgery. Nurses are in a unique position to supervise and assist patients in pain treatment, considering the extensive time nurses spend with the patients when compared to other healthcare 71professionals. Nurses are involved in assessing pain, administering analgesics, and monitoring the effect of medications (Ho et al. 2009). There is a gap between &lsquo;saying and doing&rsquo;, despite adequate knowledge about core issues in post-operative pain management. Often this is not practiced in clinical settings (Dihle et al. 2006).</div>\r\n<div>\r\n	Intravenous Patient-Controlled Analgesia (PCA) which depends primarily on self-administration of analgesics for relief has become a routine method of post-operative pain management. Studies confirmed that PCA, has proven efficacy, is convenient in providing rapid onset of analgesia and high rate of patient satisfaction (Chumbley &amp; Mountford 2010; Viscusi &amp; Schechter 2006). It is essential to assess suitability of this method of pain relief for the individual patient before commencing PCA. PCA is suitable for patients who have moderate to severe acute pain and who have been educated to use it in a healthcare setting where nurses also have been trained in its management (Chumbley &amp; Mountford 2010).</div>\r\n<div>\r\n	The ability to autonomously and immediately treat pain has been shown to reduce pain-related anxiety and stress among patient post-operatively. The use of PCA have been associated with better pain management, faster recovery of activities and shorted length of hospital stay (Herr et al. 2011; Hudcova et al. 2006). Inadequate post-operative management can negatively influence surgical outcomes, and may lead to chronic pain (Guo et al. 2012; Chumbley &amp; Mountford 2010). Patient&rsquo;s ignorance and lack of proper knowledge related to pain management may further increase barrier to obtain and optimize pain control. This lack of knowledge may influence how often they could request medication and PCA allowable medication dose (Palmer &amp; Miller 2010).</div>\r\n<div>\r\n	The Acute Pain Service (APS) team in Hospital Tuanku Jaafar Seremban (HTJS), treats nearly 800 to 900 patients who undergo surgery every month. The orthopedic patients account to 300 cases (40%). Although, patient-controlled analgesia (PCA) has been used at HTJS, there is paucity of data related to patients from different cultures who experience the same intensity of pain but may not report or respond to it in the same way. A structured educational programme regarding PCA, would be able to improve the effectiveness of pain control, patient&rsquo;s comfort and satisfaction.</div>\r\n<div>\r\n	The present study was designed to provide pre-operative PCA education to orthopedic patients in the proper usage of PCA, and to evaluate the effect of such educational programme in patient satisfaction on PCA following orthopedic surgery.</div>\r\n<div>\r\n	MATERIALS AND METHODSDESIGN</div>\r\n<div>\r\n	A pre-test post-test interventional study design was conducted in the Orthopedic units at Hospital Tuanku Jaafar Seremban (HTJS) with the intervention group provided with a structured educational programme pre-operatively on PCA using a flip chart and brochure. On the other hand, the control group received a conventional education on PCA using the Acute Pain Service (APS) protocol.</div>\r\n<div>\r\n	PARTICIPANTS</div>\r\n<div>\r\n	The recruitment of respondents was from scheduled elective orthopedic surgery, where a pre-anesthesia assessment was performed by the Anesthesiologist. Respondents who fulfilled the inclusion criteria were recruited and prescribed on the PCA for post-operative pain management. A total number of 62 respondents were recruited and the response rate was 87% with 8 respondents being excluded (transferred to other units, admitted to Intensive Care Unit (ICU) post-operatively, discharged sooner than 3 days post-operatively, communication problem due to language and cancelled surgery). In total, 54 respondents scheduled for orthopedic operation with patient control analgesia (PCA) were recruited for the study with informed consent and were divided equally into two groups (intervention and control) (n=27 in each group).</div>\r\n<div>\r\n	DATA COLLECTION AND INSTRUMENTS</div>\r\n<div>\r\n	The research instrument consisted of socio-demographic data. Pain intensity was measured using a numerical rating scale (NRS) for patient self-reporting of pain from 0&ndash;10, based on an unpleasant sensory and emotional experience associated with actual or potential tissue damage or described in terms of such damage by International Association for the Study of Pain, (IASP) and lastly a Revised (APS-POQ-R) for quality improvement of pain management consisting of 21 items scored using 0 to 10 scales. The questionnaire was piloted in clinical sites in local Malay language which has been validated (Rothaug et al. 2013).</div>\r\n<div>\r\n	STRUCTURED EDUCATION PACKAGE</div>\r\n<div>\r\n	The structured educational package on the Patient Controlled Analgesia (PCA), was adapted from St John of God Hospital with permission obtained. It consisted of a flip chart and brochure in English, was translated to Malay version by a Surgeon from Department of Surgery, Universiti Kebangsaan Malaysia Medical Centre and translated back by an Anaesthetist from Department of Anaesthesiology and a Family Medicine expert from International Medical University (IMU).</div>\r\n<div>\r\n	ETHICAL APPROVAL</div>\r\n<div>\r\n	The research was approved by the Ethics committee of UKMMC (FF-2013-475), Director of Hospital Tuanku Jaafar Seremban (HTJS) and National Medical Research Register (NMRR). The privacy and confidentiality of each individual was maintained and the respondents were given the rights to withdraw from participation.</div>\r\n<div>\r\n	DATA ANALYSIS TECHNIQUE</div>\r\n<div>\r\n	Data analysis for this study was done using Statistical Package for Social Science (SPSS) version 22. The respondents&rsquo; NRS and Revised (APS-POQ-R) score before and after intervention were both analyzed using Wilcoxon Rank Sum test to compare the mean rank as the data was not normally distributed. The relationship between socio-demographic profiles with pre-test and post-test NRS and Revised (APS-POQ-R) were analyzed by inferential stastics of Mann-Whitney test.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	The socio-demographic data was tabulated (Table 1). The total number of respondents were 54 and there were equally divided in both arms (27 respondents as intervention group and 27 respondents as control group). The mean age of all respondents was 31.96 +14.68 yrs, respondents in intervention group were younger than those in the control group (29.85 + 15.07 vs 34.07 + 14.26 ). There was equal distribution of all respondents according to age groups; below 31 yrs old (48.1%) and above 32 yrs old (51.9%). Majority of the respondents in both groups and in total were males (27.8% in control group and 37.0% in intervention group). More than half (61.1%) of the respondents were married, and this accounted to 19 respondents in the control group and 14 respondents in the intervention group. Malay respondents formed 46.3% of the study population, whereas 53.7% were Chinese and Indians.</div>\r\n<div>\r\n	LEVEL OF PAIN SCORE AMONG THE STUDY RESPONDENTS</div>\r\n<div>\r\n	Table 2 showed the level of pain score among the study respondents. Three time points were analyzed i.e. 2 hrs, 6 hrs and 24 hrs following the surgery. All patients had moderate to severe pain in the first 2 hrs following surgery. Six hrs following surgery, about 37.0% of patients had no pain to mild pain, and this included 4 patients in the control group and 16 patients in the interventional group. Majority of the respondents however, still had moderate pain to severe pain (63.0%) and this accounts for 23 patients from control group and 11 patients in the intervention group. Following 24 hrs of surgery, majority of the respondents (83.3%) had no pain to mild pain and only 16.7% of the respondent&rsquo;s experienced moderate pain to severe pain. Twenty-five respondents from intervention group and 20 patients from control group experienced no pain to mild pain, whereas 2 patients and 7 patients from intervention and control groups, respectively had moderate to severe pain, 24 hrs following surgery.</div>\r\n<div>\r\n	DIFFERENCES IN LEVEL OF PAIN SCORE AMONG THE STUDY RESPONDENTS</div>\r\n<div>\r\n	The difference in the level of pain score among the study respondents&rsquo; medians for control group at 2 hrs, 6 hrs and 24 hrs following surgery were 7.00 (IQR=3.00), 5.00 (IQR=2.00) and 3.00 (IQR=2.00), respectively. On the other hand, medians for intervention group at 2 hrs, 6 hrs and 24 hrs following surgery were 6.00 (IQR=2.00), 3.00 (IQR=1.00) and 1.00 (IQR=1.00), respectively. Significant differences in median of pain score between intervention and control group at 2 (U=142.0, p&lt;0.05), 6 (U=150.50, p&lt;0.05) and 24 (U=120.00, p&lt;0.05) hrs following surgery were tabulated (Table 3). It could be concluded that pain level at 2, 6 and 24 hrs following surgery in the intervention group was significantly lower than the controlled group.</div>\r\n<div>\r\n	LEVEL OF PATIENT SATISFACTION BETWEEN CONTROL AND INTERVENTION GROUP IN TERMS OF PAIN SEVERITY</div>\r\n<div>\r\n	The findings of this study reported the respondents&rsquo; level of pain severity with the median for the least pain in the&nbsp;first 24 hrs in control and intervention groups being 3.00 (IQR=2.00) and 2.00 (IQR=2.00), respectively (Table 4). For worst pain following first 24 hrs, the median for control and intervention groups were 8.00 (IQR=3.00) and 5.00 (IQR=2.00), respectively. The median scores for severe pain in the first 24 hrs were 40.00 (IQR=20.00) in the control group and 20.00 (IQR=20.00) in the intervention group. There were statistically significant differences (p&lt;0.05) in the medians of patient&rsquo;s pain severity at all pain levels i.e. least pain, worst pain, and severe pain between intervention and control group (least pain, U=219.50, p&lt;0.05; worst pain, U=117.0, p&lt;0.05; severe pain, U=49.0, p&lt;0.05). These findings showed that the intervention group had significantly lower pain severity following 24 hrs of surgery, at all pain severity level tested (least pain, worst pain, severe pain) compared to the control group.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This study reported respondents&rsquo; level of pain of 2 hrs, 6 hrs and 24 hrs following surgery and in the intervention group, it was significantly lower than the control group. The trend showed more respondents in the intervention group to have lesser pain than the control group, progressively through the hours. In terms of pain outcomes during the first 24 hrs, pain levels decreased in both groups from 2 hrs post-surgery to 6 hrs, and from 6 hrs to 24 hrs, post-surgery. The changes in pain level over time across both groups showed better trend in pain reduction, indicating the impact of educational intervention on pain outcomes during the first 6 hrs. The results of the present study confirm that the participants in the intervention group experienced better pain control than the control group participants for the first 24 hrs, post-surgery. The findings of the present study were consistent with previous studies (McNicol et al. 2015; Lovell et al. 2010). The findings indicated the positive effect of educational intervention on pain management following orthopedic surgery (Ho et al. 2015; Hong &amp; Lee 2012). The results also revealed that structured pre-operative teaching, using a booklet and allowing time for the learner to ask questions for clarification, is an effective teaching method. The use of written and verbal communicationreinforces the information provided. When only verbal instructions are used, information could be misunderstood by the learner. This would not be a problem with written information but when only written instructions are used, the learner may not understand the information presented if time is not allowed for questions to be answered. Also, better pain control observed in the intervention group could be related to the amount of analgesic used, which was influenced by the pain belief held by each individual.</div>\r\n<div>\r\n	In the present study, the effect of pre-operative structured education about Patient-Controlled Analgesia (PCA) on post-operative pain between the control and intervention groups concluded that pain level at 2 hrs, 6 hrs and 24 hrs following surgery in the intervention group was significantly lower than the control group. Hence, this study deemed that the intervention group was seen to be an effective mode of training for the use of PCA in terms of pain scores and were consistent with findings from previous studies (Ho et al. 2015; Park et al. 2006). This was in contrast with other studies that stated that there were not significant improvement in pain scores and outcomes (Guo et al. 2012; Lovell et al. 2010). Despite the mixed findings from previous reported studies, the current findings obtained proved the effectiveness of the structured pre-operative education programme used in this study as it has achieved expected outcomes-improvement in pain score among the intervention group. This may be attributed to more effective teaching or demonstration method to the patients on PCA use, compared to the earlier studies with negative findings. It may be concluded that patients felt better informed and less confused after pre-operative written and video presentation on the use of PCA, which directly showed the effect on patient&rsquo;s pain level.</div>\r\n<div>\r\n	The effects of pre-operative structured education programme on the level of patient satisfaction were evaluated by comparing control and intervention group in terms of pain severity, interference with function and activities, affective experience, side effects, perception of care. The present study showed that the intervention group had significantly lower pain severity, interference with functions and activities, and affective experiences; and higher perception of care compared to the control group. However, in terms of side effects, the intervention group had equal side effects compared to the control group. This study indicated that the implementation of pain education noticeably increased satisfaction scores among the patients compared to the satisfaction scores in the control group. This result was similar to previous studies (Hong &amp; Lee 2012). The results was similar to a past study which showed that systematic teaching was an effective method of improving patients&rsquo; satisfaction on the use of a PCA suggesting that the current intervention improved patient&rsquo;s pain severity, interference with function and activities, affective experience, side effects, perception of care (Hong &amp; Lee 2012). Hence, an educational programme is indeed effective in improving satisfaction with pain management. This study expands the current knowledge with regard to the effect of structured pre-operative education programme on pain score and level of satisfaction among patients using PCA. The present findings suggest that nurses and physicians caring for patients using controlled analgesia should provide a structured educational programme to help manage and ease the pain level post-surgery and increase the patient&rsquo;s satisfaction with pain management using PCA.</div>\r\n<div>\r\n	In addition, nurses are encouraged to emphasize pre-operative teaching in their practice. This information is very important for patients who are going to have surgery, as it gives them the opportunity to have control of the situation. Furthermore, it is important for health providers to consider patient satisfaction when attempting to improve adherence to pain management regimes in a clinical setting.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, the present study had a positive impact of the educational programme in managing post-operative pain and satisfaction on PCA after orthopedic surgery in a hospital. The results of this study demonstrated great implications towards improving patient&rsquo;s satisfaction, compliance to the use of PCA and adherence to self-analgesic administration.</div>\r\n', 1),
(7297, 7288, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Hypoglycaemia is a common complication of diabetes treatment. Studies have shown high prevalence of hypoglycaemia among patients with diabetes on treatment with oral hypoglycaemic agents or insulin (Marrett et al. 2009; Murata et al. 2005). Severe hypoglycaemia is associated with a 3-fold increased risk of mortality compared to non-severe hypoglycaemia (McCoy et al. 2012). Non-severe hypoglycaemia was found to affect patients&rsquo; well-being negatively, reduce productivity, as well as increase in &nbsp;healthcare utilization (Geelhoed-Duijvestijn et al. 2013). Therefore, it is important for clinicians to assess hypoglycaemia during consultation with patients with diabetes.</div>\r\n<div>\r\n	Many patients may not realise the presence of hypoglycaemic episodes. Hence, hypoglycaemia may remain unreported. The proportion of Malaysians with Type 2 diabetes who have access to home blood glucose monitoring is also low (Mastura et al. 2007; Mafauzy 2006). Therefore, many suspected hypoglycaemic episodes were either unidentified or unconfirmed. A good tool to assess episodes of hypoglycaemia may help both clinicians and researchers to identify hypoglycaemia so that treatment can be tailored, accordingly. There are very few questionnaires which were designed to measure symptoms of hypoglycaemia.</div>\r\n<div>\r\n	The Hypoglycaemia Symptom Rating Questionnaire (HypoSRQ) is a newly developed and validated tool to detect hypoglycaemia among patients with diabetes (Taylor et al. 2013; Taylor 2012) It is a valuable tool because it is one of the very few tools that demonstrated concurrent validity with blood glucose monitoring. The HypoSRQ has been found to be highly reliable with a Cronbach alpha of 0.90. The scores of the HypoSRQ demonstrated significant moderate correlations (r=0.42-0.66, p&lt;0.05) with continuous blood glucose monitoring.</div>\r\n<div>\r\n	Weinger et al. (1995) used a 40-item Mood and Symptoms Questionnaire and neuropsychological tests to determine the association between hypoglycaemia symptoms with varying levels of blood glucose. From cluster analysis, the Mood and Symptoms Questionnaire was found to have 5 cluster symptoms, which included &ldquo;autonomic symptoms&rdquo;, &ldquo;feeling weak and dizzy&rdquo;, &ldquo;feeling relaxed&rdquo;, &ldquo;negative moods&rdquo; and &ldquo;positive moods&rdquo;. However, there were 17 symptoms which did not fit into any of these clusters. All 5 symptom clusters were present when blood glucose was 2.2mmol/L. However, patients were found to be inaccurate in estimating their own blood sugar based on symptoms (Weinger et al. 1995).</div>\r\n<div>\r\n	Jaap et al. (1998) validated another questionnaire among elderly diabetic patients who were treated on insulin. The patients were asked to rate 22 symptoms of hypoglycaemia on a 7-point Likert scale. Construct validation of the responses revealed 3 components, which are &ldquo;impaired coordination and articulation&rdquo;, &ldquo;neuroglycopenic symptoms&rdquo;, and &ldquo;autonomic symptoms&rdquo;. However, there was no concurrent validation with blood glucose controls. The Cronbach alpha value for each component was unreported, as well.</div>\r\n<div>\r\n	Henderson et al. modified the Edinburgh Hypoglycaemia Scale and validated it on insulin-treated patients with Type 2 diabetes (Henderson et al. 2003). The Edinburgh Hypoglycaemia Scale consisted of 20 symptoms of hypoglycaemia. Respondents were required to rate the intensity of the symptoms on a 7-point Likert scale. Construct validation revealed 2 factors which were &ldquo;autonomic symptoms&rdquo; and &ldquo;neuroglycopenic symptoms&rdquo;. Again, there were no concurrent blood glucose measurements.</div>\r\n<div>\r\n	Brod and her team developed the Diabetes Symptom Measure (DSM) which consisted of 30 items comprising signs and symptoms of diabetes in adults (Brod et al. 2011). The DSM has good internal reliability (Cronbach alpha 0.95). Construct validation revealed 4 components which were &ldquo;high blood sugar&rdquo;, &ldquo;low blood sugar&rdquo;, &ldquo;neuropathy&rdquo; and &ldquo;general&rdquo;. The scores were significantly correlated to scores from another validated tool, the Problem Areas in Diabetes (PAID).</div>\r\n<div>\r\n	Vexiau developed an 11-item questionnaire to measure symptoms of hypoglycaemia among diabetic patients (Vexiau et al. 2008). The 11 symptoms included sweating, confusion / disorientation, shakiness, clumsy or jerky movements, dizziness, sudden moodiness or behaviour changes, hunger, tingling sensations around the mouth, difficulty concentrating, headache, and pale skin colour. Respondents were required to rate these symptoms according to severity (mild, moderate or severe) over the past 6 months. Unfortunately, construct validation was not done. There was no comparison with actual blood sugar levels as it was based on recall. However, the scores were found to be significantly associated with fear of hypoglycaemia and lower quality of life.</div>\r\n<div>\r\n	In summary, the Hypoglycaemia Symptom Rating Questionnaire (HypoSRQ) has much potential as it was validated to detect hypoglycaemia among diabetic patients alongside blood glucose monitoring (Taylor 2012; Taylor et al. 2013). In order to use the HypoSRQ in the Malaysian setting, it needs to be translated and culturally adapted for use in our local setting (Hambleton &amp; Patsula 1998). The present study reports the process of cross-cultural adaptation and linguistic validation of the original English version of the HypoSRQ into Malay and to linguistically validate both the Malay and English versions in our local setting.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a linguistic validation study conducted between January 2014 until June 2015. The permission to conduct linguistic validation on the HypoSRQ was obtained from the copyright holder of the original questionnaire, Health Psychology Reesarch (HPR). The validation process was done with close support from HPR. The linguistic validation process was guided by the recommendations of the ISPOR Task Force for Translation and Cultural Adaptation (Wild et al. 2005). Ethical approval to conduct the study was obtained from Medical Research and Ethics Committee of UKMMC.</div>\r\n<div>\r\n	TRANSLATION AND ADAPTATION PHASE</div>\r\n<div>\r\n	The original English version of the HypoSRQ underwent two independent translations into Malay language by a certified translator and an academic clinician who was a native speaker of Malay language. The translator and the clinician were requested to produce a conceptually equivalent Malay version. The two Malay versions were then compared and reviewed by the research team for suitability of the wordings. A detailed report with comments was prepared and clarifications were provided regarding reasons for the choice of words to be used in the Malay version. Amendments were made for terms that were felt inappropriate or difficult to understand by the members of the research team who were native Malay speakers.</div>\r\n<div>\r\n	Subsequently, a harmonised Malay version was sent to another two translators for back-translation into English. The selected translators were native speakers of English and had good command of Malay Language. One of the back-translators was provided by the HPR. The back-translation versions were reviewed and compared again by the research team and HPR. Comparison was made with the original English version to ensure that the Malay version had maintained semantic and conceptual equivalence. Based on the discussions, the research team agreed on a final Malay version of the HypoSRQ for the cognitive debriefing process.</div>\r\n<div>\r\n	At the same time, the research team also reviewed the original English version of the HypoSRQ for wordings that may not be familiar in the local setting. Again a detailed report was prepared and any amendments were clarified. The pre-finalised versions were submitted for review by a clinical psychologist and the comments were taken into consideration for modification and finalisation of the final version.</div>\r\n<div>\r\n	COGNITIVE DEBRIEFING PHASE</div>\r\n<div>\r\n	The final Malay version (HypoSRQ-My) and the adapted English version (HypoSRQ-EMy) of the HypoSRQ were then prepared for cognitive debriefing to determine their comprehensibility and face validity. This was done by conveniently recruiting a number of patients with Type 2 diabetes mellitus who received their treatment from a teaching primary care clinic, and a couple of patients from a paediatricendocrine clinic. Patients from the paediatric endocrine clinic were adults who were on follow-up for Type 1 diabetes mellitus since young. Care was taken to ensure a variety of patients were selected based on ethnicity, educational status, and type of therapy taken for diabetes mellitus (diet, oral antidiabetic agents or insulin).</div>\r\n<div>\r\n	The patients were invited to participate in a cognitive debriefing process and were given a separate appointment for the process. This was to give ample time for cognitive debriefing. The patients were seen individually by the researchers. Each patient was given a copy of the HypoSRQ-My or HypoSRQ-EMy to be completed. The time taken to complete the questionnaire was documented for each patient. Subsequently, a structured interview was conducted to test their comprehension of the wordings of the questionnaire. Their comments and suggestions were also recorded.</div>\r\n<div>\r\n	Based on the comments provided by the respondents, the research team reviewed the wordings of the HypoSRQ-My and HypoSRQ-EMy. The reports were reviewed together with HPR and discussions were made to draw on a consensus on the best version for local use. Wordings or phrases that were found difficult to understand by the patients were amended to improve clarity. The final version of the HypoSRQ-My and HypoSRQ-EMy were then agreed upon.</div>\r\n<div>\r\n	FORMATTING AND PROOFREADING</div>\r\n<div>\r\n	With the help from HPR, the finalised versions of HypoSRQ-My and HypoSRQ-EMy were proofread to check for errors in spelling and wording. With the help of HPR, tt was then formatted to ensure the layout conformed to the format of the original HypoSRQ. RESULTSTRANSLATION AND ADAPTATION PHASE</div>\r\n<div>\r\n	Two forward translations of HypoSRQ were compared. Comments were obtained from the translators following receipt of the translated work. In general both forward translators found some difficulty in translating certain terms into Malay language. This was because some of the terms were technical and the Malay terms were actually borrowed from the English language. While the borrowed terms existed in the approved Malay language dictionary, they were not commonly used in everyday conversation by Malaysians and thus would be difficult to understand by those with lower educational status.Consensus was achieved after the discussions (Table 1).</div>\r\n<div>\r\n	COGNITIVE DEBRIEFING PHASE</div>\r\n<div>\r\n	We recruited a total of 7 participants for cognitive debriefing of the HypoSRQ-My and 5 participants for the HypoSRQ-EMy.The characteristics of the participants were displayed in Tables 2 and 3.</div>\r\n<div>\r\n	The cognitive debriefing process was fairly straightforward. There were very few terms that required amendment after the cognitive debriefing process. Table 4 showed the terms that were particularly commented on by the participants for the cognitive debriefing of the HypoSRQ-My.</div>\r\n<div>\r\n	For the cognitive debriefing of the English adaptation, there were no difficulties overall, except for the term &lsquo;uncoordinated&rsquo;. Participants&rsquo; explanation of their understanding ofthe term showed that they had difficulty in grasping the concept of &lsquo;uncoordinated&rsquo;.Taking into consideration the findings of the cognitive debriefing of the Malay version, the team decided to drop the term &lsquo;uncoordinated&rsquo; and retain &lsquo;unsteady&rsquo;. An instruction for item no. 20 was also added to standardize both the Malay and English versions.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The aim of this project was to produce a cross-culturally valid Malay and English versions of HypoSRQ for local use in Malaysia. The Malay language is the first language of the country and is taught formally in the national education curriculum. Since English is also a widely used in Malaysia, we also aimed to culturally adapt the original English version for local use.</div>\r\n<div>\r\n	Some words used in the local English version had to be changed for better clarity without altering its original meaning or affecting the constructs being measured. The translation process into Malay and the adaption process for the local English version were generally not problematic except for few, for which obtaining a non-technical Malay term without altering its meaning was difficult. Of all the 19 full statements in HypoSRQ, only 11 terms required some discussions among the panel. In adapting the original version for local use, we had made 3 changes. This included: (i) adding a brief explanatory phrase &ldquo;(low blood sugar)&rdquo; after the word &ldquo;hypoglycaemia&rdquo;; (ii) adding minor details to increase the clarity of instruction and (iii) changed a phrase &ldquo;felt sick&rdquo; to &ldquo;nauseated&rdquo;. Cognitive debriefing was fairly straightforward and we managed to test the participants&rsquo; understanding of the Malay and English versions. There were very minimal amendments following the cognitive debriefing process, which confirmed that the translation process had been carried out well. The participants were able to show understanding of the original intended concepts of the translated and adapted version. This shows that the original HypoSRQ was well constructed with easily understood terms used.</div>\r\n<div>\r\n	HypoSRQ has been translated into various languages including Turkish (Turkey), Portuguese (Brazil) and Spanish (Peru, Mexico, USA, Columbia), and&nbsp;American English (Health Psychology Research Ltd, 2014). So far, none of the translation and cultural validation process have been published. Hence, the ease of translations and validation could not be compared. Also to the best of our knowledge, there has not been any published study regarding the utility of the HypoSRQ as yet. Therefore, although the development of HypoSRQ was rigorous, the clinical application of HypoSRQ and its various translated versions need to be assessed further by looking at its impact on clinical outcomes such as impact of intervention using HypoSRQ on quality of life of patients and metabolic control (Taylor 2012; Taylor et al. 2013). The ease of our translation and adaptation process suggests the positive value of HypoSRQ in managing patients with diabetes.</div>\r\n<div>\r\n	STRENGTH AND LIMITATION OF THE STUDY</div>\r\n<div>\r\n	The main strength of this project was the successful collaboration between the local study team and HPR. The protocol of translation and adaptation was agreed upon and adhered to strictly. Appointment of translators met the criteria set and was agreed by both study teams. Two native Malay speakers proficient in English command undertook the forward translation. One of them has been providing professional translation service and has certification in written English at master degree. The other forward translator has been an academic primary care doctor with certified proficiency by IELTS. Further, the backward translation was undertaken by at least one native English speaker who is proficient in Malay and had resided in Malaysia for few years. The other backward translation service was provided by HPR. We did not require many cycles to discuss and reconcile the final HypoSRQ-My from forward and backward translation cycles. The translated version of HypoSRQ-My was reviewed by a clinical psychologist and four clinicians before cognitive debriefing with patients.</div>\r\n<div>\r\n	A good spectrum of patients was selected with different treatment regimes, educational, socio-economic background to optimise the face validity of the questionnaire. We only had one cycle of cognitive debriefing as the understanding of terms used were easy by all participants. On the other hand, not all words in the sentences were clarified with the patients. We limited to all words or phrase that were highlighted in bold and had conceptual meaning in 19 statements like, &ldquo;dizziness&rdquo;, &ldquo;palpitation&rdquo; and &ldquo;lost consciousness&rdquo;. Almost all patients were able to provide examples of situations they experience those symptoms. We only interviewed seven and five patients for HypoSRQ-My and HypoSRQ-EMy respectively. Although small in numbers, we believe further debriefing with more patients would not result in further gain.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The original HypoSRQ was successfully adapted to HypoSRQ-EMy and translated to HypoSRQ-My. The next step in validating HypoSRQ would be to undertake psychometric evaluation of the tools in local context, before it is applicable to clinical usage.&nbsp;</div>\r\n', 1),
(7300, 7291, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Electroconvulsive therapy (ECT) was introduced by Ugo Cerletti in 1938 (Passione 2004). It is an effective treatment for major depressive disorder (MDD) (UK ECT Review Group 2003) and it is used for rapid stabilization of severe MDD patients who are highly suicidal, catatonic, psychotic, or with comorbidities that make pharmacological treatment less tolerable (Lamprecht et al. 2005). ECT is generally a safe procedure with mortality rates of 0.19 deaths per 10,000 ECT treatments (Popeo 2009). Nevertheless, to date there is limited literature regarding the safety of ECT in very low body mass index (BMI) patient. Here, a case of use of ECT in a severe MDD patient with life-threatening low BMI is reported.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	The patient was a 55-year-old Malay housewife with underlying hyperthyroidism in euthyroid state. She presented to psychiatric service for four months history of major depressive episode after her late husband passed away. She was characterized by depressed mood, anhedonia, psychomotor retardation, severe anorexia and tremendous weight loss. She also had nihilistic delusion, thinking that her internal organs ceased to function and she no longer existed in this world. To add to it, she had second person auditory hallucination commanding her to kill herself. There were persistent thoughts of ending her life, but she was too weak to attempt suicide.</div>\r\n<div>\r\n	She was admitted for one month and treated with oral fluoxetine, which was optimized until 40 mg daily. In view of poor response to pharmacotherapy and the need of rapid stabilization due to deteriorating physical health, she was transferred to another hospital with psychiatry service and ECT facility. Upon transfer, the patient&rsquo;s depressive symptoms and psychotic symptoms persisted. Examination revealed an alert, dehydrated cachexic Malay lady who appeared older than her age. Her BMI was only 11 (weight 25kg, height 152.5cm) with no hypothermia, hypotension or bradycardia. She was guarded and not fully cooperative. Prominent psychomotor retardation was noticed. Her speech was not spontaneous but relevant and coherent with reduced in tone, volume, rate, and amount. She was depressed and her affect was appropriate, non-reactive and restricted. She had active suicidal idea, nihilistic delusion, and second person auditory hallucination that commanding her to commit suicide. She was oriented to time, place, and person.</div>\r\n<div>\r\n	Initial investigations showed hypokalemia (3.1 mmol/L) and hyponatremia (120 mmol/L) with normal calcium (2.34 mmol/L), phosphate (1.23 mmol/L), and magnesium (0.83 mmol/L) level. Her hemoglobin level was 11.5 g/dL with hematocrit of 35.1%. She had hypoalbuminemia (31 g/L) while other liver function parameters were normal. Her urine culture and sensitivity showed positive growth of Morganella morganii. CA 125 was slightly raised at 40 U/ml (normal range: 0-35). Other tumor markers (CEA, Alpha-fetoprotein, CA 19.9 and Beta-HCG) were within normal range. Computed Tomography (CT) scan of thorax, abdomen, and pelvis revealed an incidental finding of right adnexa cystic lesion which was suggestive of right ovarian cyst; while CT scan of the brain showed small hypodensity in the left anterior limb of the internal capsule.</div>\r\n<div>\r\n	She was diagnosed with MDD with mood congruent psychotic features, malnutrition, electrolyte, right ovarian cyst, and hyperthyroidism in euthyroid state. Multidisciplinary approach was used to manage this patient. Her vital signs, oral intake, parenteral fluid, and urine output were monitored closely. Electrolyte imbalance was corrected and suicidal caution was activated. Complete bed rest was initiated. Intravenous Cefotaxime 1 g twice daily was started for the urinary tract infection and tablet carbimazole 10 mg once daily was continued. The patient was also given peripheral total parenteral nutrition (pTPN) with potassium supplement for one week by the Nutrition Support Team. Her fluoxetine was changed to oral escitalopram 10 mg nocte which was optimized further to 15 mg nocte.</div>\r\n<div>\r\n	ECT was initiated on day five of admission after preoperative assessment from the anesthesiology team. It was done three times a week, on alternate days. General anesthesia induction agent used was propofol while a muscle relaxant agent was suxamethonium. Her initial charge was 64mC and good electroencephalogram (EEG) seizure was recorded. For her second ECT, charge was increased by 50% to achieve supra seizure threshold. Subsequent ECT charge was gradually tapered up by 10% if seizure duration was less than 15 seconds. A total of nine therapeutic ECTs were carried out and patient was able to achieve full remission. Hence ECT was stopped. Upon discharge, the patient&rsquo;s weight was 31.9 kg with the BMI of 13. Appetite was very good and electrolytes level had returned to normal. During subsequent follow ups, the patient remained in full remission with oral escitalopram 15 mg nocte and could maintain a steady weight gains.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The patient&rsquo;s severe depressive and mood-congruent psychotic state resulted in the reduction of her BMI to a life-threatening level. Hence, rapid stabilization was necessary. As the patient had severe anorexia, peripheral TPN was used to achieve the steady weight gain at a safe rate in order to avoid refeeding syndrome, a serious complication that may occur during initial phase of nutritional support. The aim of weight gain was 0.5 to 1 kg/week in inpatient setting while monitoring the potassium, calcium, phosphate, and magnesium level closely.</div>\r\n<div>\r\n	Refeeding strategy alone was inadequate as the patient&rsquo;s anorexia state was caused by her MDD. Hence, ECT was given as a measure of rapid stabilization. Pre-operative assessment was carried out carefully as the patient had multiple comorbidities that increased the risk for anesthesia, i.e. hyperthyroidism, severe malnourishment, and electrolyte imbalance.</div>\r\n<div>\r\n	Callari et al. (2012) treated a patient with catatonia in bipolar disorder and severe comorbidities successfully with ECT. That patient had BMI of 15, deep-vein thrombosis, pressure ulcer, and systematic infections. With support of ICU care and medical treatment, including antiplatelet therapy, the patient underwent ECT, uneventfully. Poutanen et al. (2009) gave maintenance ECT for a patient with anorexia nervosa with low BMI (ranging from 14.8 to 15.3) that had comorbidity of MDD. No complication were observed during throughout the ECT.</div>\r\n<div>\r\n	From the experience encountered in this case, it is safe to conduct ECT on a patient with life-threatening low BMI that has MDD in order to achieve rapid improvement. Nevertheless, involvement of different disciplines such as anesthesiology and nutrition support team is necessary.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(7303, 7294, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Genito-pelvic penetration disorder is now inluded in DSM-5 and represents the merging concept of vaginismus and dyspareunia, which are highly correlated (Ishak &amp; Tobia 2013). In the previous classification of DSM-IV-TR (American Psychiatric Association 2000), vaginismus is classified as a sexual dysfunction and is included in sexual pain disorder category. Vaginismus is defined as, &ldquo;recurrent or persistent involuntary spasm of the musculature of the outer third of the vagina that interferes with sexual intercourse&rdquo; and the muscle spasm is described as readily observable and in some cases, as &lsquo;so severe or prolonged as to cause pain&rsquo;. In contrast, gynecological examinations have found that vaginal or pelvic spasm occurs only in few cases in vaginismic patients (Reissing et al. 2014). Primary vaginismus occurs when penetrative sexual intercourse had never happened and this is referred as &lsquo;unconsummated marriage&rsquo;. We highlight a case of young Malay women with significant anxiety and depressive psychopathology, secondary to her primary vaginismus.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	Madam Z, a 31-year-old Malay female, Assistant Auditor, married for two and half years, presented with painful sexual intercourse since she got married two and half years ago. Patient was first referred to us by gynaecology team on 22nd December 2014, which was 6 months ago for further management of her complaint. She was previously well and unaware of her condition until she got married two and half years ago. She first experienced the pain during the first time she and her husband having sexual activity. Initial stage of the sexual activity was going on smoothly. She was aroused after half an hour of foreplay, which usually involved kissing over face, neck and breast, followed by biting over neck, arm and thigh. Then, the husband inserted his tongue into patient&rsquo;s vagina. Once both of them aroused, husband then positioned her for penetration process. However, upon an attempt of penile penetration, she started to feel the pain when husband&rsquo;s penis touched the surface of her vagina. She described the pain as burning sensation and as if pain where her skin was tearing. Husband then stopped the attempt after he saw her in much pain. She then noted that there was minimal blood spotting on the bed sheet. She denied her vagina was too dry during that attempt. In fact, her vagina was lubricated well prior to penetration of husband&rsquo;s penis. Although an attempt of penile penetration and fingering caused patient to have pain, on the other hand, husband managed to insert half of his tongue in her vagina without patient experiencing any pain. She also denied having any marital conflict or argument with husband prior to that. Following that, she experienced pain over her vagina for two to three days, especially during urination and also whenever she wanted to wash her genitalia.</div>\r\n<div>\r\n	They still were having sexual activity regularly about 2-3 times/week. Whenever they had the sexual activity, her husband will still attempted for penile penetration but each time he attempted for it, patient had the pain. Pain worsened if the husband continued to attempt for it. Husband usually stopped the attempt immediately as he felt pity for her. Even though the husband stoped the attempt, the pain did not go away. Although their sexual activity was disturbed, husband never asked her to masturbate for him or doing oral sex, but there was time when he would fulfill his orgasm by rubbing his penis over patient&rsquo;s abdomen and ejaculate over it. Sometimes, husband spread the semen over her vagina with the hope that the sperm may penetrate through her vagina. She still had sexual desires and her libido was maintained. She still got aroused and excited if her husband approached her. She never avoided having sex or refused sex. At times, she was the one who started to approach her husband. Both of them never gave up in attempting for penile penetration, even though it eventually failed. She also complained that she was a bit slow to get aroused as compared to her husband. She claimed usually she required half an hour of foreplay before she got aroused. In addition, she also imagined someone else doing sex like activity in movies for her to speed up her sexual arousal parallel with her husband. She claimed she would get aroused faster if she is on top of her husband. She noticed her vagina would get lubricated easily whenever she was excited. On the other hand, her husband did not have any problem with sexual arousal. He was able to achieve penile erection after five minutes of foreplay, and able to maintain it. She was not satisfied with their sexual activity, but she could not help it because of intolerable pain but she did not feel sexual activity was a burden to her due to her limitation. She also felt pity for her husband and wished she could satisfy him. Lately, for the past 2 weeks, she had felt anxious about her sexual performance. She had occasional palpitations, feelings of unable to relax and difficulty in concentration. She also had anxiety symptoms like jittery feeling when thinking of to let her husband to penetrate her when both were ready for sex. She had the fear that the husband might get fed up or give up on her despite her husband being very supportive of her. She also had a low mood as she would be &ldquo;not a good enough wife,&rdquo; as she was depriving her husband from sexual satisfaction. She had feeling of anhedonia, negative thinking about her capacity of a good wife and sometimes felt worthlessness. She denied any deterioration of function at work. She denied history of being harassed sexually or being molested either during present or even during childhood. There was also no history of direct trauma to her genitalia. There was no history of fall or injury.</div>\r\n<div>\r\n	Patient attained menarche at age of 14 years. Her menses usually lasted for 7 days, and her menstrual cycle was regular within 28 to 30 days. She only experienced mild dysmenorrhea during first day of menses. Her menstrual flow was normal. There was no menorrhagia nor blood clots. She usually used 5 pads/day. The family tree was summarized (Figure 1).</div>\r\n<div>\r\n	Patient was 9th out of 10 siblings. She had 4 brothers, 7 sisters, and 1 stepsister. Her father passed away at age 70 years, when the patient was 6 years old, due to infection of the stomach. She claimed among sisters, she was the one who was closest to him. From what she could recall, she always spent time with her father. She described her father as very loving and nice person. Father never physically or sexually abused her. Her mother was a 68-year-old lady, still alive and healthy. Currently, she is staying with her youngest daughter. Patient described mother as a nice mother, not too strict but she was not so close with mother. She completed her primary and secondary education. She then further completed her study in Diploma in Accountancy at a public university, and subsequently further with Degree in Accountancy at the same University. She successfully graduated on time and got her job in the current workplace.</div>\r\n<div>\r\n	Physical examination and genital examination revealed normal findings. Mental examination revealed young Malay women within her 30&rsquo;s. She was been thin built with Body Mass Index (BMI) of 19kg/m2. She applied light make-up and appeared neat and tidy. She had satisfactory hygiene. She was noted to be depressed and anxious when talking about her sexual insufficiencies.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Vaginismus is probably one of the common sexual dysfunctions and its true prevalence among the general population is still unknown. This medical disorder significantly affects the quality of life and marital harmony (Frank et al. 1976). In our case, the lady presented with significant anxiety and depressive psychopathology that may further complicate her vaginismus as she was unable to perform the normal sexual intercourse activity. A specific therapy is needed to address her medical and psychological problem.</div>\r\n<div>\r\n	These therapies can help to address any underlying psychological issues, such as anxiety and combat any irrational or incorrect beliefs that one may have about sex and if necessary, be used to educate one about sex. Treatment like behavioural therapy may include systematic desensitization along with insertion of graded dilators/fingers (McGuire &amp; Hawton 2003). The sex therapy sessions include sex education, homework assignment on certain sexual practice and cognitive therapy intervention in combination of relaxation therapy. Pharmacological interventions have included short term use of anxiolytics. There is limited evidence from case series and emperical sudies.</div>\r\n<div>\r\n	In the present case discussed, a biopsychosocial approach should be based on PLISST model (Annon 1976) involving sex education (permission and limited information), graded insertion of fingers (specific suggestions), Kegel&rsquo;s exercises and cognitive behavior therapy. The use of serotonin-selective reuptake inhibitors (SSRI) may aid the therapy if patients suffer from clinical depression. The successful approach towards managing vaginismus in a clinical setting, depends on many factors such as patient, partner and motivation.</div>\r\n', 1),
(7306, 7297, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Stridor in children with subglottic cyst is rare. It is one of the potential life threatening conditions seen in the Emergency Department (ED), which jeopardize the airway. An acquired subglottic cyst is a condition resulting from previous trauma to glottic mucosa during intubation (Agada et al. 2006). Its clinical manifestations vary considerably from being asymptomatic to an airway obstruction and respiratory failure. Therefore, high index of suspicion is the key to early identification for prompt intervention from multidisciplinary teams. We highlight an important but an under-recognised case of acquired post-intubation subglottic cyst during a neonatal period presenting with stridor. The challenges encountered for identification and management of this case are being discussed.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 6-month-old baby girl, presented to ED of an academic hospital at day 3 of illness for worsening stridor with cough, rhinorrhea and poor oral intake. However, she had no fever. She was born prematurely at 27 weeks. Her birth weight was 855 gms and required ventilation support at 10 hrs of life for respiratory distress. The process of intubation was uneventful. She was initially extubated after 3&nbsp;95days but required repeat intubation for recurrent apnoeic episodes due to sepsis for another 3 days. She was noticed to have stridor since the age of 4 months, and was diagnosed for acute bronchiolitis at 5 months of age. During initial assessment, she appeared irritable. She was in respiratory distress with evidence of chest recession, and biphasic stridor, loud on inspiration. She appeared pink, and well perfused with capillary refilled time of less than 2 secs. Her respiratory rate was 56/min, blood pressure was 90/50 mmHg, heart rate was 196/min, afebrile, and oxygen saturation under room air (SpO2) was 85 %, which picked up to 100% on supplemental oxygen of 1l/min. There was transmitted sound over the lungs and reduced air entry at both bases. Other systemic examination was unremarkable. Laboratory findings showed that the total white cell count (WCC) was 9.3 x 109/l (normal range, 4-10x109/l), neutrophils 35.7%, lymphocytes 58.1%, haemoglobin 12.1g/dl (12-15 g/dl), and the platelet count was 334 x 109/l (150-410 x 109/l). Blood electrolytes were normal except that there was evidence of hyperkalaemia with serum potassium of 5.9 mmol/l (3.5-5.0). Her C-reactive protein was not raised &lt;0.003 mg/dl (&lt;1.0). Capillary blood gases on room air were as follows: pH 7.34, PaCO2 59.6 mmHg, PaO2 44.6 mmHg, HCO3 29.3 mmol/L. The infant was treated at the critical area as moderate to severe croup. However, she did not respond to intravenous dexamethasone at 2.5 mg and 2 mg of inhaled budesonide. A bolus intravenous adrenalin (1:1000) was delivered to her as she became progressively breathless and the stridor became worsened by the next few hours. She was then transferred to the Paediatric Intensive Care Unit (PICU). The patient was subjected to intubation due to worsening respiratory acidosis (Capillary Blood Gases: pH 7.25, pCO2 60.6 mmHg, pO2 61.3 mmHg, HCO3 22.5 mmol/l). However, there were difficulties with the intubation attempts. The vocal cord appeared oedematous with an unexplained resistance, which prevented the tube from passing thorough the trachea. She was urgently sent to the operating theatre for an emergency advance airway procedure. A direct laryngoscopy showed subglottic cyst (Figure 1). Subsequently endolaryngeal microsurgery (ELMS) was performed in emergency setting. Histopathological report showed benign epithelial cyst. Intravenous dexamethasone was continued for 9 days in tapering fashion. With all the efforts made, she was discharged well after 10 days of admission.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Subglottic cysts can be either congenital or acquired and can present as single or multiple cysts, but it is often difficult to distinguish, as most paediatric patients coming in with respiratory distress generally requires endotracheal intubation based on clinical judgment without prior endoscopy. The first-reported case was in 1968 with the patient&rsquo;s death due to subglottic cysts (Wigger &amp; Tang 1968).</div>\r\n<div>\r\n	The challenges remain in diagnosing subglottic cysts as early detection and intervention play a crucial role in improving a patient outcome and reducing the need for tracheostomy. A subglottic cyst clinical presentation varies depending on the size and location of the cyst. Inspiratory stridor and expiratory stridor suggest airway obstruction above and below the glottis, respectively. Biphasic stridor on the other hand, suggests of glottic or subglottic lesions. In the present case, the premature infant presented with biphasic stridor. Apart from that, she had a previous neonatal history of intubation. This condition should prompt the clinicians to narrow down the possible cause of stridor and to look for possible underlying cysts in infants&rsquo; larynx.</div>\r\n<div>\r\n	A review from previous literature showed that the development of subglottic cysts has a strong association with past history of neonatal intubation (Johnson et al. 2005). However, the duration of intubation has limited value in predicting the risk of development of subglottic cyst. A study showed that it could develop even after a period of intubation of less than 24 hrs (Johnson et al. 2005). A majority of the cases developed in premature infants (Lim et al. 2003). Agada et al. (2006) found 7 cases of subglottic cyst in a large cohort of 12,240 premature newborns. All 7 cases had history of intubation with an average intubation period of 20.6 days. The development of subglottic cyst is not always associated with history of prematurity and previous intubation. Aksoy et al. (2012) found in their study that 6 out of 9 children had no history of previous intubation.</div>\r\n<div>\r\n	Direct laryngoscopy is accepted as a gold standard tool to diagnose subglottic cysts (Zawadzka-Glos et al. 2009). In this patient, direct laryngoscopy was done on the next day when the infant&rsquo;s condition deteriorated and endotracheal intubation faced difficulty. Subglottic cysts are treatable as they can be decompressed by needle puncture and aspiration. It can safely be removed with laryngeal microdebrider with a low recurrence rate (Ransom et al. 2009). However, long-term follow-up should be planned for patients treated for subglottic cysts as there is still risk of recurrence (Lim et al. 2003).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In the present case, we highlighted the need to have a high index of suspicion to diagnose subglottic cyst in premature infant with biphasic stridor and previous history of neonatal intubation. Early diagnosis is vital to successfully manage the airway without complications.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors thank the doctors, paramedics and administrative support staff in the ED, Universiti Kebangsaan Malaysia for the assistance.</div>\r\n', 1),
(7309, 7300, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Traditional medication has a vital role in every Asian household. It is widely used due to the false perception of its safety profile as it is from herbal origin. It is also is easily available over the counter in this region from pharmacies, Chinese medical shops and convenient stores, without doctor&rsquo;s consultation or prescription. In Malaysia, there is a high prevalence (about 70%) of traditional and complementary medication usage to treat illnesses and maintain general well-being (Siti et al. 2009). Traditional Chinese medications have been identified to cause both systemic and localized adverse reaction (Papafragkakis et al. 2016 &amp; Sen et al. 2010). Although localized reactions are generally mild, rarely, severe reactions warranting hospital admission have been also been reported (Niggemann &amp; Gruber 2003). We present a case report on a patient who developed contact dermatitis after applying Chinese traditional ointment as a remedy for joint pain.</div>\r\n<div>\r\n	CASE REPORT</div>\r\n<div>\r\n	A 65-year-old Chinese woman presented with sudden onset swelling at the dorsal aspect of her right hand, area below the thumb, associated with pruritus and burning sensation. One day earlier, she had applied traditional Chinese ointment under occlusion to the joint pain at the base of her thumb. After 12 hours, she experienced redness and burning sensation at the site of application of the ointment. The following day, the redness worsened and multiple vesicles of different sizes were noted. A milder reaction had occurred a few years back when she applied the same ointment on the dorsum of her right feet. At that time she experienced minimal erythema which improved spontaneously after a few days. She had no personal or family history of atopy or any other dermatological disorders. She did not have any other medical problems except for hypertension, which was well controlled with medication. Other topical ointments and liniment methyl salicylate were not reported to cause any untoward reactions in the past.</div>\r\n<div>\r\n	Examination revealed a 4 x 5 cm, well defined erythematous patch with localized oedema limited to the area of contact with the ointment. A few vesicles and bullae were seen on the medial border of the lesion (Figure 1). Joint movements of the hand were unaffected.</div>\r\n<div>\r\n	The fluid from the bullae was drained using a sterile needle without removing the roof of the lesion. She was treated with oral prednisolone (20 mg) a day, which was tapered over the next 12 days. Good recovery was noted after a week and she was managed with topical emollient, moderate potency topical steroid of and oral antihistamine. Lesion improved after with treatment and there was complete recovery after 3 weeks, leaving a mild post-inflammatory hyperpigmentation at the affected site (Figure 2).</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The traditional medication which was applied consisted of a brownish ointment which is easily available over the counter in most Chinese medical shops. The product information displayed a list of six different herbs, menthol and camphor as the ingredients. Menthol may cause contact allergy when in contact with skin or buccal mucosa presenting as chelitits or localized irritation (Wilkinson &amp; Beck 1994; Foti 2003). Rare incidents of allergic contact dermatitis to camphor in ear drops and topical ointments were reported in the past (Stevenson &amp; Finch 2003; Noiles &amp; Pratt 2010).</div>\r\n<div>\r\n	Topical traditional Chinese medications have been implicated to cause a variety of cutaneous reactions such as urticaria, allergic and irritant contact dermatitis. (Wong 2002; Sen et al. 2010) Contact dermatitis is an inflammatory reaction of the skin due to contact with certain allergens which triggers a complicated pathway of inflammatory mediators. It is commonly classified as irritant and allergic contact dermatitis. Irritant contact dermatitis arises as a result of direct inflammation to chemical or physical stimuli while allergic contact dermatitis arises as a result delayed contact hypersensitivity reaction caused by allergens. Both these conditions are closely linked and clinical differentiation between irritant and allergic contact dermatitis is sometimes difficult as both may co-exist together. The morphology of this adverse reaction may vary from mild erythema, vesicles, bullae or erosions in severe cases. Factors affecting the severity of the lesions include the concentration of the product, extent of application, duration of exposure and host susceptibility. Personal or family history of atopy increases the risk of developing contact dermatitis due to</div>\r\n<div>\r\n	Figure 1: Right hand showing a well-defined erythematous patch with vesicles of varying sizes on the medial edge of the lesion.</div>\r\n<div>\r\n	Figure 2: Photograph showing right hand after 3 weeks of treatment. A small area of post-inflamatory hyperpigmentation WAS noted at the site of the previous lesion. the presence of abnormal skin barrier function (Ale &amp; Maibacht 2010).</div>\r\n<div>\r\n	In the present case, it was unknown whether the adverse reaction produced is due to the herbal, menthol or camphor component in the ointment as the patient refused further testing using patch test. Establishing the specific allergen responsible for the allergic reactions is challenging due to the possibility of content adulteration with prescription drugs, unintentional contamination or misidentification of herbs. Intentional contamination or nondisclosures of product contents by the manufacturers are the other hurdles in identify possible allergens. The possibility of new allergen formation as a result of interaction between the ingredients within the product cannot be ruled out either. A recent study in Singapore inferred that the presence of essential oils in topical traditional Chinese medications may be responsible for the adverse cutaneous reactions seen with the use of these products (Sen et al. 2010).</div>\r\n<div>\r\n	One of the important steps in managing contact dermatitis is to prevent recurrent episodes. This can be done by identifying and eliminating the involved allergen. Hence, patient education and advice on avoidance of triggers are appropriate preventive measures. Sensitivity or patch testing may be done for suspected cases as it is useful in differentiating contact dermatitis form other morphologically similar dermatological disorders. The mainstay of treatment for acute contact dermatitis is topical corticosteroids. More severe reactions may benefit from a short course of systemic corticosteroid. Primary lesions complicated by secondary bacterial infection may require additional treatment with antimicrobials. Emollients and barrier creams applied to the affected skin may confer additional benefits and aid in the recovery process (Bourke 2001).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Physicians and consumers should be aware that traditional medications can potentially cause adverse reactions. A detailed history of traditional medication use must be probed as this information may not be voluntarily given due to the misconception that these products are safe. Contact dermatitis secondary to traditional medication should be considered as a differential diagnosis in patients presenting with well defined eczematous lesions, involving skin overlying a joint or in poorly healing wounds. Physicians must also be aware of the possibility of cross sensitization resulting in a similar reaction with the use of other topical traditional medications. Identifying the specific triggers and future avoidance would help eliminate recurrent episodes.</div>\r\n', 1),
(7313, 7304, '<div>\r\n	Patient positioning is a vital step in preparing a patient for surgery. The aim of positioning of a surgical patient should ensure comfort, dignity, and allow good access to the operative site, while minimizing risk of injury to the patient (Knight &amp; Mahajan 2004). Certain surgical procedures require specific positioning as wrong positions may interfere with the surgical technique resulting in intra-operative technical difficulty.</div>\r\n<div>\r\n	Patient positioning may be regarded as a pertinent constituent of surgical planning (Knight &amp; Mahajan 2004; Rozet &amp; Vavilala 2007). The ideal patient position involves balancing surgical comfort to both the surgeon and patient (Rozet &amp; Vavilala 2007). Among the usual apparatus used to position patient is an air bag placed beneath the shoulders for a hyperextended neck, mainly in cleft lip &amp; palate repair, and head and neck surgeries. We discuss how certain surgical positions achieved via re-using items readily available in operating theatres.</div>\r\n<div>\r\n	A 60-year-old Malay male, with no known prior medical illness presented with an anterior chest wall swelling, gradually increasing in size for the past 10 years. Initially, the patient was asymptomatic until recent six months during which he started to exhibit constitutional symptoms of malignancy such as of weight loss. Otherwise, the only complaint was the anterior chest wall mass. On examination, the mass was 6 by 4 cms, oval in shape, firm, mobile and not pulsatile. There were no superficial skin changes.</div>\r\n<div>\r\n	Computer Topographic (CT) imaging showed a large hetero-genous enhancing and lobulated anterior mediastinal mass with local invasion to the sternum and ribs. Imaging also showed the right thyroid gland was enlarged with a large hypo-dense lesion, coarse calcification present within the lesion. The thyroid mass extended from the level of C4 vertebra down to the level of the clavicle, displacing the trachea laterally to the left, conserving the trachea patency. Numerous sub-centimetre mediastinal lymph nodes were present. Subsequent fine needle aspiration cytology (FNAC) was highly suspicious of underlying thyroid follicular carcinoma.</div>\r\n<div>\r\n	Multi-disciplinary team was assembled to tackle the highly suspicious of thyroid malignancy, anterior chest wall mass and for the reconstruction and wound closure of the huge anterior chest wall defect. Intra-operative positioning of the patient involving different surgical disciplines needs different patient positional requirements. This is to accommodate for an adequate surgical field exposure, sufficient area for the anaesthetist to maintain homeostasis and minimizing risk of injury to the patient. Reusable items which are readily available in the operation theatre were modified as inflatable air bags was used to position patients accordingly before the operation begins. Firstly, a 3 l saline irrigation bag was procured once its fluid contents were emptied and dried out. Then, a stopper was placed in one tube while the other end to a 3-way valve via a suction tubing (Figure 1). A second piece of suction tubing connected the 3-way stopper to a manually controlled sphygmomanometer bulb (Figure2).</div>\r\n<div>\r\n	Post-operatively, patient recovered from surgery without major complications; not from patient positioning and preparation. Input from oncology, rehabilitation and physiotherapy was obtained to enhance patient recovery.</div>\r\n<div>\r\n	Surgical positioning before surgery is a vital part of surgical planning. Different surgical disciplines in general, have their own preference so as to know which position is best and ideal for performing certain procedures. Head and neck surgeries usually prefer a hyperextended neck to get good access to the anatomically tight neck area and avoid hindrance from other surrounding structures mainly the shoulders. Orthopaedics specialities such as spinal instrumentations require patients to be in prone position as it is almost impossible to get access from the anterior aspect of the body.</div>\r\n<div>\r\n	Appropriate surgical patient positioning is not only important for the surgeon but to the supporting staff as well, especially for the anaesthetist and the circulating staff to prevent serious adverse events and complications (Rozet &amp; Vavilala 2007). Patients are under general anaesthesia and are unable to care for themselves or signal medical staff of impending injuries. Among common complications from poorly positioned patients during surgery, it includes peripheral nerve injuries, ocular injuries, and pressure sores (Knight &amp; Mahajan 2004).</div>\r\n<div>\r\n	With reference to the case discussed, access to the neck is crucial in view of small access to the neck area and the grossly enlarged thyroid gland which is highly suspicious of malignancy. Adequate exposure will assist the surgeons to dissect the area better, identifying vital structures more easily as well as able to resect structures better without injuring the surrounding structures (Wisner &amp; Jacoby 2004).</div>\r\n<div>\r\n	To achieve an adequate hyperextended neck, few techniques have been described; mainly via using sand bags or inflatable air bags (Wisner &amp; Jacoby 2004). This apparatus is placed beneath the shoulder at the level of the scapula blades, raising the body slightly allowing the neck to be extended to a certain degree. Advantages of using a sand bag is that the sand bag area is usually better in fixing the body to the same position but not easily adjustable as sand bag usually comes in a fixed shape and volume (Wisner &amp; Jacoby 2004; Collins et al. 2012). Air bags on the other hand uses inflated air into a bag placed behind the shoulder (similar to the sand bag). Air bags settings can be modified more readily by adjusting the amount of air inflated. However, air is compressible and therefore patients&rsquo; position could alter with certain movements during the surgery (Wisner &amp; Jacoby 2004; Collins et al. 2012).</div>\r\n<div>\r\n	Another advantage of using an air bag is the apparatus is easily replicable and reproducible via items readily available in the operation theatre. For this case, a used (empty) normal saline bag was connected to a hand pump as diagram showed above. The empty bag was placed beneath the shoulders and inflated accordingly to achieve the desired position required. This can be recycled and reproducible as these items are usually thrown away after use and could cut cost massively. There is no need for maintenance for this in house designed air bag as the hand pump can be stored easily in the OT cabinet whereas the used bag is almost always available since fluid is always used by anaesthetist in OT.</div>\r\n<div>\r\n	This air-bag is not only used for head and neck and chest surgery but also in positioning our cleft lip and palate children during their cleft repair procedures.</div>\r\n<div>\r\n	Surgical patient positioning is important aspect of the surgical planning and necessary to determine the surgery success. There are multiple modalities and ways to position the patient to the surgeons&rsquo; desire, but financial constraints is a major issue in healthcare nowadays. In-house inflatable air bag is a creative way of positioning a surgical patient and an evolution to cut healthcare cost worldwide.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to thank the General Operating Theatre Unit of Universiti Kebangsaan Malaysia Medical Centre for their contribution.&nbsp;</div>\r\n', 1),
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(67602, 67588, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Honey, a part of ancient medicine, has recently become the key target of attention for treating certain diseases, promoting overall health and well being. Honey is known as natural sweet substance made from honey bees from floral nectar. Honey is commonly used to treat many ailments (Wen et al. 2012). Since ancient times, various types of honey and their derivatives are being used to treat several diseases such as cancer, infections, delayed wound healing, etc (Hussein et al. 2012). The knowledge on traditional usage of honey has been verbally passed through generations to generations. Thus, honey draws attention of many researchers. The information on the therapeutic values of honey was aimed to proceed with further researches. Consequently, it stimulated the several scientific studies on specific honey to verify the claims of community people on their medicinal value.</div>\r\n<div>\r\n	Gelam Honey (GH) is one of Malaysian wild monofloral honeys produced by Apisdorsata bees. The main nectar as well as the pollen collected by the bees are from the plant named Melaleucacajupati Powell or locally known as the &ldquo;Gelam tree&rdquo;. The plant belongs to the Myrtaceae family. GH is produced in large amounts in the state of Terengganu on the eastern coast of peninsular Malaysia, where these mangrove trees grow abundantly (Moniruzzaman et al. 2013a). GH is usually prepared by base hydrolysis and extracted with ethyl acetate (Wahdan 1998). Locally in Malaysia, the timber of gelam is known as kayu putih and has a tall evergreen tree of 24 meter height. In Malaysia, the species of Melaleuca grow naturally in swamp forests behind the sandy beaches and the mangroves in the states of Kedah, Melaka, Negeri Sembilan, Kelantan and Terengganu. The trees are well recognised by their characteristic thick papery flaky bark (Lim &amp; Midon 2001). The common English name for Gelam tree differs from place to place and often known as the &ldquo;swamp tea-tree&rdquo; and also referred to as the &ldquo;paperback tea-tree&rdquo; and the &ldquo;cajeput&rdquo; tree (Ng 1978). The purpose of present review is to provide the current therapeutic information of GH and its usage in medical research along with complete overview of its phytochemistry, traditional uses, and pharmacological values. In addition, the present review summarise evidence-based findings on GH and its therapeutic properties, which will help to propose future research prospects.</div>\r\n<div>\r\n	TRADITIONAL USES OF GELAM HONEY&nbsp;</div>\r\n<div>\r\n	In Malaysia, oral administration of GH is used to treat the cholera. Its topical application is for the treatment for thrush, vaginal infection, acne, athlete&rsquo;s foot, verruca, warts, insect bites, cold sore and nits. In Myanmar, GH is purified from the leaves to treat gout. The Indochinese uses cajuput oil for rheumatism and applied as analgesic whereas the oil is used externally in Indonesia for burns, colic, cramps, earache, headache, skin diseases, toothache and wounds. In Philippine, the leaves are used to treat asthma.When administered orally, it induces sweating and acts as antispasmodic (Koh et al. 2009). The papery barks of Gelam trees are used for caulking wooden boat (Lim &amp; Midon 2001).</div>\r\n<div>\r\n	PHYSIOCHEMISTRY AND CHEMICAL COMPOSITION OF GH</div>\r\n<div>\r\n	A physiochemical property of honey is very important as its characteristic contribute to the production of quality honey. The physiochemistry of GH from the previous studies was shown in Table 1 (A-Rahaman et al. 2013). The pH value of GH was acidic in nature. The pH value represents the stability against microbial spoilage, life expectancy of honey product and fermentation process (Silva et al. 2013; Bogdanov 1997). GH was reported to have dark colour with Pfund scale of 139.00 mm. The color of GH was categorized as light amber to dark amber (The National Honey Board 2003). The colour of GH was due to the presence of some pigments such as chlorophylls, carotenoids, flavonoids and derivatives of tannins and polyphenols (Juszczak et al. 2009). GH was reported to have high moisture content (up to 20%). The moisture content denotes the quality and shelf-life of honey product. The factor of storage combined with the high moisture content in honey increase the fermentation rate, and result in formation of acidified honey. Moreover, the moisture content of honey highly depends on climatic change, maturity of honey and its harvesting season (Finola et al. 2007). A group of researchers observed that the moisture content indicates the origin of honey. In a tropical country like Malaysia with rainy season all over the year leads high moisture content in GH. Thus, Malaysian honey is always treated by evaporation to reduce the water content and to increase the quality.&nbsp;</div>\r\n<div>\r\n	Phytochemical composition of Gelam Honey</div>\r\n<div>\r\n	Honey was reported to have a broad range of chemical composition more than 200 ingredients, including sugars, amino acids, vitamins, minerals, polyphenols and enzymes (Bogdanov et al. 2008). Various studies were conducted to identify the nutritional and non-nutritional constituents such as enzymes in GH (Khalil et al. 2011) (Moniruzzaman et al. 2013b) (Moniruzzaman et al. 2014). Table 2 showed the summarized chemical composition of GH.</div>\r\n<div>\r\n	ANTIOXIDANT PROPERTIES OF GH</div>\r\n<div>\r\n	The term of &lsquo;oxidative stress&rsquo; describes an imbalance between cellular production of reactive oxygen species (ROS); generation of free radicals and the counter acting antioxidant protective activity in a certain organism (Ames et al. 1993; Moylan &amp; Reid 2007). Oxidative stress disorders results from inability of endogenous antioxidants to neutralize the abnormally high level of ROS. The increased level of ROS destroys the complex cellular molecules such as lipids, proteins and DNA (Matough et al. 2012). Cells are naturally equipped with antioxidant defence systems to counter balance free radicals production and protect against oxidative stress through an interacting network of antioxidant enzymes. ROS such as hydroxyl radical (OH&bull;), superoxide anion (O2&bull;), and hydrogen peroxide (H2O2) are naturally generated in the body during normal metabolism. They are neutralized by endogenous antioxidant enzymes such as superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidise (GPx), to prevent oxidative stress (Betteridge 2000). Although, aerobic organisms possess an antioxidant defence system, damage occurs when free radical production exceeds the limit of system&rsquo;s ability and this condition indirectly results in accumulation of H2O2 in the cells. Numerous chronic diseases such as cancer, cardiovascular diseases, impairs wound healing and gastrointestinal inflammatory diseases are consequences of oxidative damage. Several modern medicines are introduced in the management of these chronic diseases. The positive and effective outcomes are also achieved from such medicines. However, these medicines offer the increase risk of drug resistance and pronounced side effects. Natural products are believed to have lesser side effects in comparison with synthetic agents. These folk medicines are enriched with several therapeutic properties. They serve as a source of antioxidant and proven to improve the oxidant and anti-oxidant imbalance by reducing the ROS production (Ferreira et al. 2009). GH has been accounted to have high phenolic and non-phenolic content to attenuate oxidative damage. The antioxidant activity of GH is mainly due to the presence of phenolic acids, flavonoids, catalases, peroxides, carotenoids and non-peroxidal components.&nbsp;</div>\r\n<div>\r\n	Several experimental studies were conducted to confirm the putative anti-oxidative activities of GH on animal models. A recent study observed the protective effect of GH against oxidative stress in young and aged rats. It was revealed that supplementation with GH reduced the DNA damage and plasma malondialdehyde (MDA) level in young rats compared to the control group. GH supplementation also significantly increased the antioxidant cardiac SOD activity and cardiac catalase (CAT) activity in both young and aged rats. Meanwhile, there were no changes in erythrocytes superoxide dismutase (SOD) and glutathione peroxidise (GPx) activity in both young and aged group but CAT activity increased in young rats (Sahhugi et al. 2014). A recent study revealed the synergistic effect of GH and ginger on oxidative stress and metabolic profile in streptozotocin-induced diabetic Sprague-Dawley rats. The study claimed that combination of GH and ginger provided a potential antioxidant effect in the diabetic rats. The results showed significant reduction of SOD, CAT activities andreduced MDA level in the diabetic treated rats compared to the normal rats and diabetic control group (Sani et al. 2014). Earlier study also reported that pre-treatment with GH at a dose of 6 mg/ml decreased the DNA damage in gamma-irradiated human diploid fibroblasts (HDFs) (Hussain et al. 2014). Increase gamma-ray exposure resulted decreased cell survival rate in HDFs. However, following treatment with GH at pre- and during-radiation, increased the cell survival rate was observed. The study showed that GH acts as a protective agent against gamma-irradiation in HDFs (Makpol et al. 2012).</div>\r\n<div>\r\n	In 2013, a new study conducted to utilize an alternate model to observe the anti-oxidative effects of GH on pancreatic hamster cells (HIT-T15) under hyperglycaemic conditions. Pre-treatment of HIT-T15 cellswith GH showed a significant decrease in the production of ROS, glucose-induced lipid peroxidation, a significant increase in insulin content and the viability of cultured cells under hyperglycaemic conditions. The results proved that supplementation with GH to HIT-15 cells give highest cell viability at a concentration of 80 &mu;g/mL and 80 &mu;M, respectively. The cell viability of HIT-15 cells in hyperglycaemic status was significantly increased (p &lt; 0.05) following pre-treatment with GH compared to the cells cultured with glucose alone. In addition, the ROS production and MDA level in the both cells under normal and hyperglycaemic condition was inhibited and reduced by the pre-treatment with GH. Pre-treatment with GH extract significantly reduce (p &lt; 0.05) F2 isoprostane production. It indicated that GH decreases the damages caused by increased free radicals production in hyperglycaemic state. Moreover, the study also showed that pre-treatment with GH exhibited a significant increase (p &lt; 0.05) in insulin content (Batumalaie et al. 2013). From the above findings, it was proven that GH has both anti-oxidant and anti-diabetic properties as it increased the insulin content in hyperglycaemic state.</div>\r\n<div>\r\n	ANTICANCER PROPERTIES OF GH</div>\r\n<div>\r\n	Cancer is an abnormal growth of cells derived from a single cell, loses their normal control mechanisms and proliferates continuously by invading adjacent tissues. This is also a process of cellular and organ dysfunction. There are various types of cancer arising from different organs namely; colon, breast, lung and liver. Several studies have been conducted to observe the anti-cancer properties of GH. Combination treatment of GH and ginger extract showed synergistic effects via increasing the apoptotic rate of HCT 116 colon cancer cells and reducing the 1C50 of GH required for inhibiting the cells growth. It also showed that co-treatment of GH with Flurouracil (5-FU) has potential anti-tumour effect against colorectal cancer cell by reducing the cell growth compared to the treatment with 5-FU alone (Hakim et al. 2014).</div>\r\n<div>\r\n	Another study was also conducted to investigate the anti-proliferative effect of GH against HT 29 colon cancer. Comparatively, the result showed that GH was more potent to suppress the growth of colon cancer cells compared to nenas honey, with a lower IC50 of 39.0 mg/ml and 85.5 mg/ml, respectively. The results showed decreased in DNA damage and percentage of apoptotic cells in HT29 colon cancer cells treated with GH. It also showed significantly decreased production of prostaglandin E2 (PGE2) levels compared to H202 induced cells without treatment. Thus, the findings suggested that GH is capable to suppress the growth of HT29 colon cancer cells by inducing apoptosis and impeding inflammation (Wen et al. 2012).</div>\r\n<div>\r\n	In another study, a combination of GH and crude ginger extract was found to have potential chemo preventive effect against HT29 cells. The combined treatment stimulated early apoptosis by up regulation of caspase 9 and I&kappa;B genes accompanied by downregulation of the cancer genes. These results highlighted that combination treatment of GH with ginger has an efficient chemo preventive property in managing the cancers (Tahir et al. 2015). However, the study observed on the effect of GH alone on certain cancer cells is still lacking.&nbsp;</div>\r\n<div>\r\n	ACCELERATE WOUND HEALING PROPERTIES OF GH</div>\r\n<div>\r\n	Wound healing is a complex biological cascade of cellular and biochemical events consist of 3 phases; inflammation, proliferation and maturation (Vidinsky et al. 2006; Aljady et al. 2000). Despite recent advances in health care, inadequate wound management and development of secondary infections resulting in high morbidity is still an important concern to the public healthcare. Hence, wound management still remains a vital focus of research. Recently, the interest of using alternative therapeutic agents coming from natural sources has increased alarmingly. Honey is one of the ancient and most enduring natural therapeutic agents in the wound management (Salmah et al. 2005; Suguna et al. 1992). An experimental study showed the positive effect of GH against excisional wound on the neck of Sprague-Dawley rats. GH was applied to the wound on day 13 and compared with untreated and saline group. GH treatment accelerated wound contraction compared to commercial gel with less scar formation (Tan et al. 2012).</div>\r\n<div>\r\n	Another study observed the effect of GH on corneal keratocytes proliferative capacity and phenotypic characterization. The study focused on two different supplemented GH media which were basal media (BM) and basal media with serum (BMS). GH at concentration of 0.0015% in both media showed the highest proliferative capacity with no morphological changes compared to the control group. The corneal cell supplemented with 0.0015% of GH in the two media showed high density of growth with higher gene expression of aldehyde dehydrogenase (ALDH), quiescent keratocytes, vimentin and fibroblast compared to the control group. In contrast, the alpha smooth muscle actin (&alpha;-SMA) expression, was lower in GH treated group compared to the control group. Thus, it is believed that GH at concentration of 0.0015% promoted ex-vivo corneal keratocytes proliferation while retaining desirable phenotype expression. These results showed GH as a potential therapeutic natural agent in improving corneal wound healing (Yusof et al. 2016).</div>\r\n<div>\r\n	In addition, a group a researchers worked on the wound healing properties of GH and developed the idea of honey hydrogel dressing for the management of wound healing (Mohd Zohdi et al. 2012). It was found out that application of honey hydrogel dressing significantly increased (p &lt; 0.05) wound closure rate and the rate of re-pithelialization compared to control hydrogel and OpSite film dressing. Based on the histopathological evidence, it showed that honey hydrogel significantly attenuated inflammatory response (p &lt; 0.05) on seventh day of treatment and modulated the proinflammatory cytokines involve in wound healing at molecular level. This study has clearly demonstrated the potential effectiveness of honey hydrogel dressing in accelerating the burn wound healing through the various effects on cellular elements and cytokines involved (Mohd Zohdi et al. 2012).</div>\r\n<div>\r\n	ANTI-INFLAMMATORY PROPERTIES OF GH</div>\r\n<div>\r\n	Inflammation is a complex pathological response of the body which is classified as acute and chronic depending on the time and duration of the event (Ferrero-Milliani et al. 2006; Nathan 2002). Inflammatory process takes place in several disease such as cancer, inflammatory bowel disease, central nervous disorder or degenerative disease and autoimmune diseases. Various inflammatory markers such as interleukin 6 (IL-6), IL-12, &nbsp;tumour necrosis factor (TNF) and &nbsp;cyclooxygenase-2 are released during inflammation. These cytokines initiate and implicate the inflammatory process. Nuclear factor kappa B (NF-kB) serves as important regulator for cytokines and growth factors expression in the inflammation (Iwalewa et al. 2007). It was reported that oral administration of GH significantly inhibited the oedema at two and three hrs following carrageenan injection in a one day of pre-treatment model. However, on seventh day pre-treatment rat model, a reduction in oedema started one hr following caarrageenan injection. It showed that a longer duration of GH treatment provided the better outcomes. The treatment with GH showed a significantly decreased in the production of proinflammatory cytokines compared to the control group. A similar effect was observed in indomethacin treated group (NSAID). Plasma was collected to analyse the production of inflammatory mediators such as NO, PGE2, TNF-&alpha;, IL-6, as well asInos and COX-2 following treatment with GH. Interestingly, the production of all the inflammatory mediators were decreased in a dose dependent manner and this suggested that GH is potentially useful for treating inflammatory conditions by reducing the oedema and inflammatory markers (Hussein et al. 2012).</div>\r\n<div>\r\n	On the other hand, the same group of author had conducted another experiment to investigate the anti-inflammatory mechanism of GH in carrageenan-induced rat paw via NFkB pathway. The rats were supplemented orally with GH (1 or 2 g/kg body weight) at durationof 1 and 7 days. The expression of NFkB (p65 and p50) and ikBa genes in inflamed rat paws were significantly reduced following pre-treatment with GH. The expression of pro-inflammatory mediators such as COX-2 and TNF-alpha were found to be reduced following the treatment with GH in rat paw tissues. It is believed that GH exhibited its inhibitory effect by attenuating NF-kB translocation and inhibiting IkBa degradation, followed by decreasing the inflammatory mediators COX-2 and TNF-&alpha;&alpha;in wound tissues (Hussein et al. 2013).</div>\r\n<div>\r\n	Instead of oral supplementation, intravenous injections of GH have also proven in giving a potent effect in protecting organ from lethal doses of lipopolysaccharide (LPS). The rabbits were given intravenous injection of 1 mL honey (500 mg/kg in saline). The sample of blood and organs were collected following eight hours of LPS injection and haematological parameters, biochemical tests, histopathology and myeloperoxidase assessment were measured. It was observed that treatment with GH showed protective effects by reducing the neutrophils count and decreasing the myeloperoxidase activity. Less mortality rate was found in the treated group compared to the control group. Thus, honey may exert a potential therapeutic agent in protecting organs against toxicity of LPS (Kassim et al. 2012).&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	To date, researchers give more focus of attention to natural product that has been claimed to have various therapeutic benefits in comparison with the synthetic drugs. Natural honey is well known to have various therapeutic properties such as antioxidant, anticancer, anti-diabetic, anti-inflammatory and improved wound healing effects. Among the honeys, GH is widely used in Malaysia as a traditional medicine. The potential efficacy of GH alone or combined with other herbal extracts draws the attention of many researchers. The positive outcomes were observed following the treatment with GH. Phytochemical properties of GH and its constituents bring forth the desired beneficial effects. This review summarised the evidence-based findings from various studies including experimental and molecular researches. The present review opens the door for the scientists and medical health personnel to carry out the future researches in conjunction with the above therapeutic properties of GH.</div>\r\n', 1),
(67605, 67591, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	The metatarsals are the intermediate long bones of the foot. Metatarsal fractures denote 5-6 % of fractures faced in the health care centres (Hatch &amp; Rosenbaum 1994). Among five metatarsals, the first, second and fifth are frequently injured metatarsal bones of the foot. However, the fifth metatarsal (5th MT) bone accounts for the highest incidences of fracture in any sports. With the increase awareness of sports in general population, the number of participants is increasing alarmingly.</div>\r\n<div>\r\n	The anatomy of the 5th MT reflects its fracture pattern. The 5th MTis located at the lateral side of the foot (Dameron 1975). This bone comprises base, a shaft, a neck and a head. The tuberosity is the expanded part of the base of the bone. Fractures of the base of the 5th MT are one of the most commonest injuries of the foot (De Lee 1996). The tuberosity of the 5th MT protrudes laterally, while the tendon of peroneus brevis and the lateral band of the plantar aponeurosis are inserted to the tuberosity (Figure 1). These strong ligaments make the proximal part of 5th MT fixed while it&rsquo;s distal part relatively mobile. Hence, in fracture, it creates instability and may predispose to poor fracture healing.</div>\r\n<div>\r\n	Increased motion results from increased contraction of the peroneus brevis tendon and lateral band of the plantar aponeurosis in acute inversion of the foot lead to the fracture of the tuberosity of the 5th MT bone. This type of fracture accounts for majority of proximal fractures (Theodorou et al. 2003).</div>\r\n<div>\r\n	Tuberosity of 5th MT receives blood supply from metaphyseal vessel that penetrates the non-articular surfaces. The proximal diaphysis of 5th MT receives blood from nutrient artery. In area just distal to the tuberosity, the longitudinal branches of the diaphysis artery join the metaphyseal vessel that gives rise to the watershed area. This creates an area that relatively has poor blood supply. Hence, it is associated with poor fracture healing and may complicate with delayed union or non-union (Smith et al. 1993).</div>\r\n<div>\r\n	Three different fractures occur in the base of 5th MT bones namely zone 1, zone 2 and zone 3. Zone 1 represents the fracture that occurs at the tip of the base (tuberosity) of 5th MT. This type of fracture is also known as avulsion fracture. Zone 2 fracture or Jones fracture occurs in between the base and the shaft of the 5th MT. Zone 3 fracture occurs at the distal to the shaft of the 5th MT (Lawrence &amp; Botte 1993) (Figure 1).</div>\r\n<div>\r\n	Depending on the type of fracture that occurs in the 5th MT bone, several non-operative and operative procedures were carried out. In addition, the outcomes and complications that arose from each therapeutic intervention were also widely discussed in the earlier studies (Kavanuagh et al. 1978; Sides et al. 2006; Kelly et al. 2001). However, the detailed benefits and impediments of both procedures related to 5th MT bone fracture were not reviewed, to date. Therefore, the present review attempts to look at the factors that might influence in decision making management of 5th MT fracture.</div>\r\n<div>\r\n	SCORING SYSTEM, DECISION ANALYSIS MODELAND NEW CLASSIFICATION SYSTEM</div>\r\n<div>\r\n	Tahririan et al. (2015) established a scoring system, which can be used to weigh between operative and non-operative approaches in 5th MT fracture (Table 1). In this scoring system several factors were found to give weight towards surgical approach. These factors include type III fracture, fracture displacement more than 2 mm, weight of the patient more than 81 kg, female and the presence of diabetes mellitus as co-morbidities. The cut-off of point of the total score was 9, in which greater value favours surgical approach.</div>\r\n<div>\r\n	Decision analysis model developed by Bishop et al. (2015) reported operative fixation is the preferred management strategy for 5th MT fracture. This model was developed based on outcome probabilities and utilities. Outcome probabilities is based on data extracted from 19 studies on 5th metatarsal fracture that is one randomized controlled trial, one prospective case series, and 17 retrospective case series. Outcome utilities were based on patient preferences for various disease states obtained from 32 adults (25 women, 7 men) with no history of foot injury. The expected value for intramedullary screw fixation was 7.88 and non-operative treatment was 7.74 suggesting that operative treatment was a favourable approach. However, the expected values between operative and non-operative approaches were differed by only 0.3 on a 10-point scale, which may explain controversy surrounding decision making in the management of 5th MT fracture.</div>\r\n<div>\r\n	Attempts have been made to reclassify 5th MT fracture. Polzer et al. (2012) recommended 5th MT fracture to be classified as metaphyseal and meta-diaphyseal fractures based on prognosis and therapeutic consequences. Metaphyseal fractures are defined as fractures that do not extend beyond the distal end of the fourth-fifth intermetatarsal articulation, regardless of the number of fragments, displacement or intraarticular involvement and it is recommended to be treated functionally. Meta-diaphyseal fractures defined as fracture just distal to the fourth-fifth intermetatarsal articulation and it is recommended to be treated with intramedullary screw fixation.&nbsp;</div>\r\n<div>\r\n	NON-OPERATIVE OUTCOME</div>\r\n<div>\r\n	Displaced oblique spiral fractures of the 5th MT shaft showed excellent long-term functional outcomes when treated non-operatively with only two delayed unions and three non-union developed from 142 patients (Aynardi et al. 2013).</div>\r\n<div>\r\n	Fracture of 5th MT treated with removable walking boot and without weight bearing restriction had favourable outcome in which 24 (89%) out of 27 patients achieved clinical union at a mean of 8.0 &plusmn; 2.6 weeks with only one (4%) patient developed non-union (Marecek et al. 2016).</div>\r\n<div>\r\n	In one prospective, randomised clinical trial reported there was no significant difference in the outcome of patients with 5th MT fracture between the group of patients that received immobilisation treatment with below the knee cast and the group of patients who received symptomatic treatment with double elastic bandage (Akimau et al. 2016).</div>\r\n<div>\r\n	Furthermore, routine outpatient follow-up of 5th MT fracture has no added clinical value and can be discharged from the Emergency Department and allowed weight bearing as tolerated, provided adequate discharge advice is given (Ferguson et al. 2013). &nbsp;</div>\r\n<div>\r\n	OPERATIVE OUTCOME</div>\r\n<div>\r\n	Systematic review comparing outcome between operative and non-operative approaches reported that surgical approach is recommended in 5th MT fracture in view of lesser non-union rate and lesser time needed for union. This review includes six relevant studies with a total of 237 patients in which 49% underwent surgical intervention and 51% treated non-operatively. In all studies, intramedullary screw was the surgical approach employed to patients. The rates of non-unions were lower in operative management ranging from 0% to 11%, in comparative with non-operative management ranging from of 11% to 50%. Time to union, return to sport and normal activity was significantly lower in surgical management compared with non-surgical approach. The rate of complications were higher in non-operative approach with 31% of patients needing surgery following conservative treatment compared to only 8.5% who needed repeat surgery in operative approach (Yates et al. 2015).</div>\r\n<div>\r\n	FACTORS INFLUeNCE INTRAMEDULLARY SCREWS FIXATION</div>\r\n<div>\r\n	The unique anatomy of the 5th MT in particular, its lateral curvature makes surgical technique inheritably challenging. Ideally, in intramedullary screw fixation it should be performed with the largest screw possible, both in its length and diameter in order to provide the best possible construct (Tan et al. 2016). It is a common problem encountered during surgery when there is excessive screw length leading to lateral gapping and malreduction of the fracture (Tan et al. 2016). Considering lateroplantar curvature of 5th MT bone begins when its medullary canal starts to taper, it is recommended to keep the screw length less than 68% of the length of the 5th MT bone. This corresponds to the average straight segment length of the metatarsal bone which was 52 mm measured from its proximal end and it represented 68% of the overall length of the bone (Ochenjele et al. 2016). It is recommended to use lateral radiograph to estimate the length of the screw as distance from apex to the base is smaller in this view (DeSandis et al. 2016).</div>\r\n<div>\r\n	Accurate diametre of the screw is crucial as under sizing leads to instability of the fixation leading to failure of fixation, while oversizing increases risk of iatrogenic fracture (Scott et al. 2015; Granata et al. 2015). It is reported that 4.5 mm cannulated screw is the narrowest screw diameter that can provide adequate fixation, considering a cadaveric study that reported the mean dorsal to plantar and medial to lateral diameter of the 5th MT bone as 6.475 &plusmn; 1.54 (range 4 to 12) mm and 4.6 &plusmn; 0.85 (range 3 to 6) mm respectively (Scott et al. 2015). Radiographic study of 199 patients suggested screw size greater than 4.5 mm may provide optimum reduction considering coronal diameter of the isthmus of 5th MT bone was greater than 4.5 mm in 81% of males and 74% of females (Ochenjele et al. 2015). It is recommended to use anteroposterior radiograph to estimate diameter of the screw as the canal shape is elliptical and was smaller in this view (DeSandis et al. 2016).</div>\r\n<div>\r\n	Bicortical purchases of the screw may increase the strength of the fixation. It is reported that all patients treated with bicortical screw fixation for displaced zone I and zone II fractures healed with radiological union achieved at 5.33 &plusmn; 1.03 and 6.59 &plusmn; 1.84, respectively (Mahajan et al. 2011). Screws positioned at proper angle and perpendicular to the fracture line may help achieve bicortical purchase. In order to achieve this, an imaginary line connecting tuberosity of the 5th MT bone and third metatarsophalangeal joint can be used to position the leading guide wire. These anatomical landmarks can be assessed during surgery when the patient is put in lateral position with the hip joint neutral in position, knee joint flexed to an angle of approximately 15 and the ankle positioned in slight plantar flexion (Wang et al. 2016). In order to maintain the proper position of the screw during its placement, it is recommended to incorporate percutaneous reduction and stabilization of the fracture using pointed reduction clamp prior to screw fixation, which was reported to prevent iatrogenic displacement and gapping at the fracture site (Tan et al. 2016) (Figure 2).</div>\r\n<div>\r\n	Options for screws in 5th MT fractures are diversed in type and size. A study comparing between groups of patient treated with traditional screw and screw designed specifically to be used in 5th MT fracture reported there was no significant difference in fracture union between the two groups. However, group with traditional screw was associated with increased number of adverse events including implant failures, intraoperative fracture and symptomatic hardware (Metzl et al. 2013).</div>\r\n<div>\r\n	Complications associated with 5th MT fracture include non-union, delayed union and failure of fixation. Failure of fixature presents as refracture, malreduction and symptomatic hardware (Yates et al. 2015).&nbsp;</div>\r\n<div>\r\n	Refracture following operative treatment was reported in 7.3% cases which occurred within eight months. Failure occurred in patients treated with smaller diametre of cannulated screw suggesting proper selection of screw size may reduce risk of refracture especially in athletes (Granata et al. 2015).</div>\r\n<div>\r\n	Pre-treatment of non-union with pulsed electromagnetic fields (PEMFs) at non-union site together with open reduction and internal fixation significantly shortened union time to 8.9 weeks in comparison to 14.7 weeks for group without PEMF (Streit et al. 2016).</div>\r\n<div>\r\n	OTHER SURGICAL METHODS</div>\r\n<div>\r\n	Mini-hook plate has been designed so that its hook provides compression force, rotational stabilization and grasping effect of the comminuted fragments. It is an effective alternative surgical hardware for zones I and II 5th MT fractures with displaced or comminuted small fragments. Fracture union with mini hook plate was reported at a mean of 54 &plusmn; 11 days (range, 38-74 days) and returning to premorbid daily activities at a mean of 74 &plusmn; 10 days (range, 63-98 days) (Choi et al. 2013).</div>\r\n<div>\r\n	Locking compression plate (LCP) distal ulna hook plate was designed for fractures of distal ulna. Due to the similarity in anatomical structure between distal ulna and proximal 5th MT bone, this plate was used in 5th MT fracture. In a study involving nineteen patients, it was reported that this LCP distal ulna hook plate is suitable to be used in osteoporotic, comminuted and tuberosity avulsion (zone 1) of 5th MT fractures with average union achieved at 7.4 weeks (Lee et al. 2014).</div>\r\n<div>\r\n	Headless compression screw is a self-drilling screw that provides excellent holding power and compression effect to the fracture site. In a study involving 60 athletes treated with headless compression screw reported less adverse events and fast recovery with mean time to start running at 6.3 weeks (range, 3-12.7 weeks) and full activity following surgery at 11.2 weeks (range, 6-25 weeks) (Nagao et al. 2012).</div>\r\n<div>\r\n	Plate fixation was recommended in laterally bowed 5th MT of comminuted fracture with union achieved at mean of 56.8 days (range, 30-92 days). However, one third of the studied patients required reoperation for plate removal (Kadar et al. 2015).</div>\r\n<div>\r\n	Among competitive athletes, patients with metatarsus adductus angles, 5th MT lateral deviation, prominent 5th MT styloids and high fourth-to fifth intermetatarsal are recommended to perform bone graft as it is reported to be associated with &nbsp;reduced risk of complications (O&rsquo;Malley et al. 2016; Tsukada et al. 2012).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	Several factors may influence decision making in the management of 5th MT fractures. Outcome of previous studies, surgical technique, surgical method and appropriate hardware may influence approach in this fracture.&nbsp;</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(67610, 67596, '<div>\r\n	Over the past few decades, there is an increase in sports and sport related activities. As a result, there was also an increase in sports related injuries. &nbsp;In any sport injury, the common affected parts of the body include head, shoulder and knee. Ankle is one of the commonest region affected in any sport injuries (Fong et al. 2007). Intrinsic factors include past history of ankle sprains, anatomical variations, size of the foot, flexibility of the foot, weight of the individual as well as gender (Beynnon et al. 2002). Extrinsic factors include presence of air cells in the heel of shoes, landing surface, stretching prior to exercise and position of the players and use of equipment in any sport (McKay et al. 2001; Kennedy et al. 2005). More than 75% of the ankle injuries are lateral ankle sprains (Hopkinson et al. 1990; Andy et al. 2011; Nuhmani &amp; Khan 2013). Other than sports, individuals are known to sustain ankle injuries in motor vehicle accidents, martial art and military training (Yeung et al. 1994). Ankle injuries need proper treatment and management so that there are no residual symptoms which may keep an individual from any sport.&nbsp;</div>\r\n<div>\r\n	The aim of the present review paper was to highlight the normal anatomy of the ankle, mechanism of injuries related to sports, congenital abnormalities or anatomical variations related to ankle injury and discuss its effective management.</div>\r\n<div>\r\n	Anatomy of the ankle joint</div>\r\n<div>\r\n	The ankle joint, a unilateral axial type of synovial hinge joint is also termed as talocrural joint. The bones which contribute to the formation of this joint include inferior surface of lower end of tibia proximally and lateral surface of medial malleolus of tibia and medial surface of the lateral malleolus of fibula. Distally, these bones articulate with trochlear surface of the talus to form ankle mortise. &nbsp;The malleolus of fibula extend more distally than malleolus of tibia (Moore 2014; Floyd 2009).</div>\r\n<div>\r\n	The fibrous capsule is attached to the margins of the articular surfaces proximally and distally. The joint is weak anteriorly and posteriorly but thickened medially and laterally as it is strengthened by the collateral ligaments.</div>\r\n<div>\r\n	Ankle joint is strengthened by ligaments such as lateral and medial ligaments. The lateral ligament strengthens the lateral part of the ankle joint and it is formed by three separate bands. It is known as anterior talofibular, calcaneofibular and posterior talofibular ligaments. The anterior most ligament is anterior talofibular ligament which has a flat, weak band extending from lateral malleolus to the neck of talus. The middle band is the calcaneofibular ligament and it has a round cord, traversing from the tip of lateral malleolus to the lateral surface of calcaneus while the most posterior band is known as posterior talofibular ligament (Nancy et al. 2012; Moore 2014). It has a thick strong band, extending from the medial malleolar fossa to attach to the lateral tubercle of the talus (Nancy et al. 2012; Moore 2014). Schematic diagrams of all talofibular ligaments were drawn (Figure 1 &amp; Figure 2).</div>\r\n<div>\r\n	The medial side of ankle is protected by medial ligament. It is also known as deltoid ligament. It has four strong, large ligamentous bands which attach proximally to the medial malleolus, then spread out to attach distally to the talus, calcaneus and navicular bone (Nancy et al. 2012). These four ligamentous bands are known individually as anterior and posterior tibiotalar, calcaneotibial and tibionavicular ligaments. The deltoid ligament is crossed by tibialis posterior and flexor digitorum longus muscles.&nbsp;</div>\r\n<div>\r\n	Muscles related to ankle joint anteriorly are tibialis anterior, extensor hallucis longus, extensor digitorum and peroneus tertius. These muscles together with anterior tibial vessels and nerve are bound together deep to extensor retinaculum. Posterolateral to the ankle joint are peroneus brevis and peroneus longus tendons. The structures identified behind medial malleolus of tibia (arranged in sequence from before backwards) include tibialis posterior tendon, flexor digitorum longus muscles, posterior tibial vessels and nerve, and flexor hallucis longus.&nbsp;</div>\r\n<div>\r\n	The talocrural joint is supplied by malleolar branches of fibular anterior and posterior tibial arteries while the lymphatics drain via veins accompanying the arteries and into long and short saphenous veins. It is innervated by the tibial nerve and deep fibular nerve (Moore 2014).</div>\r\n<div>\r\n	There are two types of movements which occur at the ankle joint, i.e. dorsiflexion and plantarflexion. Inversion and eversion movements are commonly thought to be occurring at ankle joint but these movements occur at subtalar joint, not the ankle joint (Lin et al. 2006).</div>\r\n<div>\r\n	Another joint located superior to the ankle joint, is the distal tibiofibular joint which is of syndesmosis type. The lower end of tibia and medial surface of lower end of fibula form this joint. These articulating bones are held together by interosseous membrane, anterior and posterior tibiofibular ligaments. It functions as stabilizer to prevent the separation of the distal end of tibia and fibula under the effect of body weight (Lin et al. 2006; Joshua et al. 2011). Anterior and posterior tibiofibular ligaments are considered as primary stabilizer for distal tibiofibular joint.&nbsp;</div>\r\n<div>\r\n	The most common ligament which is affected during injury is the lateral ankle ligaments, especially the anterior talofibular ligament, followed by calcaneofibular ligament and posterior talofibular ligament. Deltoid ligament injury is rare in occurrence. Any tear or sprain is usually associated with a broken fibula (Nancy et al. 2012). Injury to the distal tibiofibular syndesmosis (less 18%) occurs less frequently than lateral ankle injury (Hopkinson et al. 1990; Norkus &amp; Floyd 2001)</div>\r\n<div>\r\n	Mechanism of ankle injury</div>\r\n<div>\r\n	Many sites in the body are affected in sports but the most common injury is in the ankle region (Fong et al. 2007). Ankle sprains are the commonest type of ankle injury followed by fracture, strain, abrasion, contusion, tendinitis, blister, impingement, cramp, bruise and laceration (Fong et al. 2007). Among all ankle sprains, 77% involve lateral ligament injuries (Gerber et al. 1998). Athletes sustain these injuries while running on uneven terrain, stepping on other athlete&rsquo;s foot during play and improper landing from a jump. The most common reason for ankle injury is due to inversion with the foot in plantar flexed position (Baumhauer et al. 1995; Nuhmani &amp; Khan 2013).</div>\r\n<div>\r\n	Anatomically, lateral malleolus of fibula extend more distally and posterior compared to medial malleolus. This allows more inversion than eversion of the foot. Lateral ankle ligaments function to resist excessive inversion and plantar flexion of the ankle. Thus lateral ligament complex are more susceptible for inversion injury. It usually happens during increase touchdown plantar flexion. Normal range for plantar flexion is 50˚. &nbsp;A cadaveric study showed lateral ankle ligament tear with inversion of talus more than 30˚ (Joshua et al. 2011).</div>\r\n<div>\r\n	The severity of the injury occurs from anterior to posterior, involving first the anterior talofibular ligament, followed by calcaneofibular ligament and then the posterior talofibular ligament depending on the severity of the injury (Nuhmani &amp; Khan 2013). Other structures that are injured during lateral ankle sprain include peroneal tendons, joint capsule, and the proprioceptive nerve endings which are found within these soft tissue structures (Joshua et al. 2011).&nbsp;</div>\r\n<div>\r\n	The mechanism of injury in lateral ankle ligament sprains and high ankle sprains are different. Lateral ligament sprains occur while foot and ankle are in plantarflexion and inversion while in high ankle sprains happens during excessive dorsiflexion and eversion of the foot and ankle (Hopkinson et al. 1990; Norkus &amp; Floyd 2001). &nbsp;Injury to the deltoid ligaments is rare and less frequently documented (Lin et al. 2006).</div>\r\n<div>\r\n	Lateral ankle sprains are graded from 1 to 3, according to the severity. A grade 1 is considered as a mild sprain, involving microscopic tearing of anterior talofibular ligament (ATFL) but the function of the ligament is to restrain unwanted motion is not compromised. Patient may complain of minimal pain and swelling over the ligament. Examination revealed, minimal tenderness upon palpation over the affected area and patient is able to ambulate with no instability. In moderate grade 2 ankle sprains, it involves a partial tear of ATFL, but the calcaneofibular ligament remains functionally intact. The symptoms become more prominent with inability to hop, and obvious limp with walking. A grade 3 ankle sprain involves a complete rupture of the AFTL with partial or complete disruption of calcaneofibular ligament. The joint stability and function are lost (Nuhmani &amp; Khan 2013).</div>\r\n<div>\r\n	The anterior talofibular ligament is tested with the anterior drawer&rsquo;s test and the calcaneofibular ligament is assessed by the talar tilt test. Both tests are performed with the foot in slight plantarflexion (Lynch 2002, Nuhmani &amp; Khan 2013).</div>\r\n<div>\r\n	Ankle injury in the particular sport</div>\r\n<div>\r\n	The incidence of ankle injuries is reported to be high in court games and team sports such as rugby, soccer, volleyball, basketball, American football and Australian football (Fong et al. 2007). The injury could occur during landing on the surface or landing on the other players&rsquo; foot.</div>\r\n<div>\r\n	Basketball</div>\r\n<div>\r\n	Basketball is considered an aggressive non-collision sport. It involves frequent jumping, landing and body contact with other players. For that reason, ankle is a common injury occurred during basketball with 15.9% of occurrence compared to knee (10.7%), trunk (6.5%), thigh (5.4) and leg (5.0%) (Fong et al. 2007). Out of all ankle injuries, ligament sprains with incomplete tears were the most frequently diagnosed (Nelson et al. 2007). A survey was done on 10393 basketball players who participated in the elite and recreational basketball competition showed that 45% of ankle injuries incurred during landing (McKay et al. 2001). Three risk factors contributing to the ankle injury were identified, i.e. previous history of ankle injury, presence of air cell in the heel of shoes and players who did not stretch prior to the game (McKay et al. 2001). The use of external ankle support such as brace and tape was proven to reduce incidence of ankle injury especially in player with history of ankle sprain (Handoll et al. 2001; McGuine et al. 2011).</div>\r\n<div>\r\n	A study conducted on professional female basketball players showed that most of the injuries occurred inside the key area of the basketball court which accounted for 56.3% of all ankle sprains (Kofotolis &amp; Kellis 2007). Players&rsquo; positions also contributed to the risk factor in any ankle injury. Interestingly, a player who played in the centre had a higher rate of injury than players in other positions (Kofotolis &amp; Kellis 2007).</div>\r\n<div>\r\n	Football</div>\r\n<div>\r\n	Majority of the reported ankle injuries in football are ankle sprains (Fong et al. 2007). The players sustained ankle sprain during hitting an uneven surface of field or stepping on another players&rsquo; foot while running or landing from a jump. A specific mechanism which related to football is a direct contact injury, in which the opponent&rsquo;s leg slides into the other player&rsquo;s leg during tackling thereby leading to improper landing of foot on the ground (Eric et al. 2003). A study conducted on 320 intercollegiate football players at the National Football League Combine proved that position of the players has significant risk factor for ankle injury, i.e. kickers (100% incidence), special teams (100%), running backs (83%), wide receivers (83%), and offensive linemen (80%) (Kaplan et al. 2011). The other common injury after lateral ankle ligament sprain in football players is high ankle sprain or syndesmotic sprain. Up to 25% of all ankle injuries in football involve the ankle syndesmosis (Sman et al. 2014). The offensive linemen are at risk to get this type of sprains. &nbsp;Jump height and balance performance are predictors for ankle syndesmosis sprains, while age, body size, flexibility and muscle strength did not increase the risk of high ankle injury. &nbsp;However, according to Tyler et al. (2006), an overweight player who had a previous ankle sprain was 19 times more vulnerable to sustain any non-contact ankle sprain compared to a normal-weight player with no previous ankle sprain (Tyler et al. 2006). Past researchers emphasized on intervention program on proper technique on landing, falling and recovery to protect the players from this type of injury (Scase et al. 2006).</div>\r\n<div>\r\n	Rugby</div>\r\n<div>\r\n	Rugby is a tough, full contact team sport. Thus, the inherent injury risk is substantial.&nbsp;</div>\r\n<div>\r\n	Lower extremities are the most susceptible body part during training and competition (Jakoet &amp; Noakes 1998; Sankey et al. 2008; Palmer-Green et al. 2015; Whitehouse et al. 2016). Out of 416 players observed during 1995 Rugby World Cup, 42% of the injuries involved lower limb followed by 29% in upper limb and 17% on the face. Of all injuries in the lower limb, 53% of injuries occurred during tackling, 23% during the ruck and maul, 11% during open play and 9% during foul play (Jakoet &amp; Noakes 1998). According to researchers, ankle injury (9.3%) is the third commonly occurring injury following knee injuries (16.1%) and thigh (14.3%) (Whitehouse et al. 2016). However, different observations were made during Rugby World held in 2003, in which the injury rate to head and face were twice than that of the injury at the foot and ankle (Best et al. 2005). Important to note was that injury to the ankle made the professional players take a longer period to return to recover (Sankey et al. 2008).&nbsp;</div>\r\n<div>\r\n	The most common injury to ankle in rugby player is lateral ankle sprain with 25% having anterior talofibular ligament tear (Sankey et al. 2008; Kaux et al. 2015). According to earlier reports, syndesmotic injuries, ankle sprains, degenerative injuries, Achilles tendon injuries and deltoid ligament sprains were the next most common match injuries (Sankey et al. 2008).&nbsp;</div>\r\n<div>\r\n	There are intrinsic and extrinsic factors which contribute to the ankle injuries such as history of ankle injuries, forefoot varus, overpronation of foot, surface of play, position of player and wearing protective equipments (Sankey et al. 2008). Most of the injuries occurred during phase of tackling, affected either the one that tackle (tackler) or the one being tackled (ball carrier). However, ball carriers sustained a greater percentage of injuries to the lower limb compared to the tacklers (Kenneth &amp; Will 2008).&nbsp;</div>\r\n<div>\r\n	All position of player has risk to sustain lateral ankle sprain, however players in second row forward are more susceptible compare to back row forward position (Sankey et al. 2008). Syndesmotic sprain is less common in this sport; however it is more serious and requires a longer duration of absence than lateral ankle sprains (Sankey et al. 2008).</div>\r\n<div>\r\n	Badminton</div>\r\n<div>\r\n	Racquet related sports include badminton, tennis and squash. Although these games have typical similarities in terms of using racquets, net and court, the nature of the injuries sustained vary because of different playing technique and relative weight in the racquet, ball and shuttle.</div>\r\n<div>\r\n	Badminton is the popular game played worldwide. It is a non contact sport which requires constant running, repetitive jump, thrust, lunge, quick changes of direction and rapid arm movement from a wide variety of postural position (Shariff et al. 2009). This explains why lower extremities are the most affected in this game. Separate studies conducted in Malaysia, Sweden, Denmark, United States, Canada and Cambridge showed that lower limb extremities are the most affected body site during badminton compared to upper limb, back and neck (Chard &amp; Lachmann 1987; Fahlstrom et al. 1998; Kroner et al. 1990; Shariff et al. 2009; Bahareh et al. 2013). Ankle is the second most injured after knee, followed by thigh, heel and toes. Another study in Hong Kong showed that the ankle injury is the commonest body site affected in badminton. &nbsp;Mechanism of ankle injury observed in female Iranian elite badminton players were mostly during landing (28.6%), landing with rotation (28.6%) and stopping suddenly (28.6%) and toe off position (14.2%) (Bahareh et al. 2013).&nbsp;</div>\r\n<div>\r\n	Ankle injury in running</div>\r\n<div>\r\n	Running injuries are often related to either excessive motion or excessive shock. Eighty percent of running disorders are mainly due to overuse injury (Walther et al. 2005). The predominant site for leg injuries are knees followed by ankle and foot (Lun et al. 2004; Fong et al. 2007). In the study done during Hong Kong International Marathon on 580 runners, 33.9% of the injuries were in knee while 20.9% were in the region of the ankle (Purves &amp; Chan 1987). It is multifactorial involved in the injury-intrinsic and extrinsic factor. Intrinsic factor is a factor related to the anatomy of the foot, degree of pronation, flexibility of foot and weight (Novacheck 1998; Butler et al. 2006; Lopes et al. 2012). The extrinsic factors include shoes type, stretch prior to run, running surface and distance (Lun et al. 2004). Researchers emphasized for every shoes design to provide two important criteria i.e shock absorption at heel, stabilization of hindfoot motion during loading response and forefoot during stance phase (Novacheck 1998).&nbsp;</div>\r\n<div>\r\n	There are three type of injuries related to running, i.e. Achilles tendinopathy, plantar fasciopathy and ankle sprains (Lopes et al. 2012). Ankle sprains usually occur in relation with running surface. The most common ligament affected is the anterior talofibular ligament followed by calcaneofibular ligament and posterior talofibular ligament if the injury more severe (Tenforde et al. 2016). A runner who has the presence of accessory bone at the posterior aspect of talus (os trigonum) may develop os trigonum syndrome.</div>\r\n<div>\r\n	Ballet</div>\r\n<div>\r\n	It is almost impossible for ballet dancer to not injure the foot and ankle.Commonly, the injuries related to overuse and acute trauma. (Macintyre &amp; Joy 2000; O&rsquo;Loughlin et al. 2008; Allen et al. 2012; Gamboa et al. 2008). &nbsp;According to Kadel et al. (2006) 34-62% of injury involved ankle and foot. A retrospective review was conducted on 204 elite ballet dancers over 5 years period revealed 53% of injury occurred in the foot or ankle, followed by 21.6% in the hip, 16.1% in the knee, and 9.4% in the back (Gamboa et al. 2008). Lower extremity mainly affected followed by the back (Gamboa et al. 2008; Milan 1994) as extreme physical demand placed on muscles, tendon and ligament of these region. Ballet requires strength, endurance and great flexibility of the human body. &nbsp;Overuse happened due to long hours of the ballet training that involved strenuous, repetitive movements of the ankle while acute injury occurred in relation to improper technique during landing or rolling. There were many factors which contributed to the injuries such as anatomical variations of the foot, improper technique, dancing surface, frequency and duration of performance. The incidence and severity of the injuries related to intrinsic factors were greater than those of injuries related to extrinsic factors (Allen et al. 2012).&nbsp;</div>\r\n<div>\r\n	Ideally, the ballet dancer needs to perform more than 100&deg; attempts of plantar flexion. This leads to ankle injuries during poor landings from jumping, misstep while on en-pointe or demi-pointe position (Milan 1994). As the dancer&rsquo;s foot is always in hyperplantarflexion and inversion, it causes more stress on the anterior talofibular ligament (ATFL) to resist the motion (O&rsquo;Loughlin et al. 2008).</div>\r\n<div>\r\n	Common injuries involving ankle are lateral ankle sprain, posterior and anterior ankle impingement syndrome (Malone &amp; Hardaker 1990; Milan 1994; O&rsquo;Kane &amp; Nancy 2008; Niek van Dijk 2006). In lateral ankle sprain, a dancer complained of swelling, tenderness of the lateral aspect of foot with unstable foot. It happens because damage of ATFL in excessive forced inversion of a plantar flexed foot (Milan 1994). Proper rehabilitation of ankle sprains is important for early return to dance and to prevent further ligamentous injury to the ankle. It can be achieved by practising ballet exercise in the swimming pool as the buoyancy of water minimizes the weight bearing stress (Malone &amp; Hardaker 1990).&nbsp;</div>\r\n<div>\r\n	Posterior impingement syndrome occurs due to repetitive plantarflexion of the foot (Russell et al. 2010). It occurs due to the entrapment of soft tissue and bony process between inferior surface of tibia and trochlear surface of talus with superior surface of calcaneus (Milan 1994). Dancers experience pain over the hind foot, aggravated with forced plantarflexion movement during en-pointe position. If the dancer has os trigonum (anatomical variation of the hind foot in which, dancer has extra bony process at the posterior aspect of the talus) and presents with similar symptom, it is described as os trigonum syndrome (Milan 1994). In this case, the effective treatment is to remove the structure with simple surgical excision followed by an aggressive rehabilitation program. Most of the professional ballet dancers return to their unrestricted dance activity after 3 months post surgery (Marotta &amp; Micheli 1992).&nbsp;</div>\r\n<div>\r\n	Anterior ankle impingement syndrome occurs due to repetitive forced dorsiflexion of the foot especially during pli&eacute; position and landing from the jump (Milan 1994; O&rsquo;Kane &amp; Nancy 2008). Later, the recurrent traction to the anterior joint capsule will cause hypertrophy of the soft tissue in the anterior ankle joint or proliferation of the osteophytes (Tol &amp; van Djik 2004). Dancers complained of pain over the anterior ankle aggravated with loaded dorsiflexion of the foot. Upon examination, there is tenderness on palpation at the anteromedial or anterolateral joint line with or without swelling. Plain radiograph reveals the diagnosis. In the presence of spurs or osteophytes, the diagnosis is anterior bony impingement. In the absence of spurs or osteophytes, the diagnosis is anterior soft tissue impingement.</div>\r\n<div>\r\n	Anatomical consideration of foot and ankle alignment that liable for ankle injury</div>\r\n<div>\r\n	Os trigonum</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Os trigonum is an accessory bone at the posterolateral aspect of talus, which presents bilaterally (Jones et al. 1999). It develops from second ossification center which usually appear at the age 8-11 years. When it fails to fuse, one year of its appearance, it is known as os trigonum. However, it is still connected to the lateral tubercle of talus by fibrocartilaginous synchondrosis. When it is fused, it is called &lsquo;fused os trigonum&rsquo; or &lsquo;Stieda process&rsquo;. It presents approximately in 5 to 15% of normal feet (Marotta &amp; Micheli 1992). It is usually asymptomatic or noted incidentally by radiographic finding. However, it becomes symptomatic as a result of impingement of the soft tissue between the process and the posterior aspect of the tibia when the ankle is held in plantar flexion. It becomes obvious with strenuous physical activity of the lower limb or during ankle injury, and termed as &lsquo;os trigonum syndrome&rsquo;. Patient presented with posterior ankle region pain occurred mostly when pushing off on the big toe (as in walking) or when pointing the toes downward (Marotta &amp; Micheli 1992). It occurs due to the micro trauma from repetitive hyperplantarflexion or an episode of acute forced hyperplantarflexion which compress the os trigonum between posterior malleolus of tibia and posterior process of calcaneus. This syndrome commonly occurs in ballet dancing, soccer, downhill running and volleyball.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Pes cavus</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Pes cavus is also termed as high arched foot. It involves both the rear- and forefoot in which it is characterized by a high medial longitudinal arch, plantarflexed first ray, claw toes, and callous over the 5th metatarsal base. It is caused by bone malformations in the foot or nerve problems. One can develop this kind of foot if one continually wears shoes that have small toe box. Tight toe box force the toes to passively flex and raise the arch making the soft tissue structures to shorten and contract. This foot condition is very painful especially with motion (Linda 2009).&nbsp;</div>\r\n<div>\r\n	This kind of foot is ideal for dancers as the requirement for plantar flexion is maximal. However, this position will limit dorsiflexion. Hence, dancers&rsquo; are more susceptible to anterior impingement syndrome.&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Pes planus</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Pes planus is also known as flat foot arch or flat foot. It can be structural which usually present from birth. It is normal for infant and young children to have flat feet but as they grow older, the normal arch forms. However, the normal arch does not form in the few children for unknown reasons. Pes planus may also be functional, caused by loose soft tissue structures of the plantar portion of foot. An athlete with pes planus since birth, may not have any specific symptoms and signs of foot discomfort. However, they may have a tired feeling in the feet after prolonged standing, walking or running. The athlete, who has a foot injury, also may have pes planus (Linda 2009).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Management</div>\r\n<div>\r\n	&nbsp;&nbsp;</div>\r\n<div>\r\n	The principle management of acute ankle injury is R.I.C.E (rest, ice, compression and elevation). During acute situation, physical examination is not reliable as it is obscured with pain and swelling. Thus, ice therapy, non steroidal anti-inflammatory drugs, massage, compression bandage and leg elevation above the heart level are very important to ease the pain, reduce the swelling and inflammation (Chorley 2005; Joshua et al. 2011; Nuhmani &amp; Khan 2013). Traditional Chinese medicine also been used widely in China to reduce pain and swelling and promote recovery of ankle function. It includes herb, acupuncture and massage (Koo et al. 2002; Joshua et al. 2011). Later, further treatment is given according to the diagnosis.</div>\r\n<div>\r\n	The treatment for type 1 and 2 ankle sprains is mainly conservative. The use of Aircast ankle brace, elastic support bandage were recommended than immobilization. Immobilization may result in joint stiffness, loss of propioception and late return to sports (Nuhmani &amp; Khan 2013). Aircast ankle brace appear to be more effective to restore ankle joint function compared to elastic bandage support bandage (Boyce et al. 2005).</div>\r\n<div>\r\n	If the leg&rsquo;s swelling and pain is not reduced or becomes more severe after the initial treatment, there is highly suspicious of grade 3 lateral ankle sprain. Usually magnetic resonance imaging (MRI) study is conducted to assess the severity of the injury (Nuhmani &amp; Khan 2013). Few surgeons opt for operative approach, but others prefer non-operative approach as the treatment.</div>\r\n<div>\r\n	Players with syndesmotic ankle sprains are prescribed walking boots to avoid separation of the distal tibia and fibula. This allows healing of the interosseous ligaments within four to six weeks (Fong et al. 2009). Rehabilitation program post injury is very important and has been considered as a part of the management for ankle injury. The effectiveness of the program ensures the recovery of the ankle and the future performance of the athlete (Mattacola &amp; Dwyer 2002). Inadequate management of ankle sprain injury may lead to residual symptoms and chronic problems to ankle such as chronic ankle instability and osteoarthritis. It was reported that 74% of players with ankle sprain complained of persisting symptoms up to 4 years following injury. Up to 20-40% of these patients develop chronic symptoms such as synovitis, tendinitis, swelling, pain, stiffness, weakness and crepitus (Fong et al. 2009; Valderrabano et al. 2007). Epidemiology study on Hong Kong Chinese athletes, reported that the athletes with less than 5 times history of ankle sprained complained of pain as their residual symptom, while athletes with more than 5 times history of ankle sprain suffered with instability (Yeung et al. 1994).</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Prior anatomical knowledge of the lower limb, especially the ankle joint is important for ascertaining the exact nature of injury sustained in any sport. The present review was a humble attempt to discuss the ankle injuries related to sports and discuss the best available treatment outcomes.&nbsp;</div>\r\n', 1),
(67614, 67600, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Medication wastage, as defined by the Delphi technique, amongst a wide range of experts, refers to any medication, which expires or remains unused throughout the whole medicine supply chain. It also refers to the unnecessary consumption of medications by patients, or the unjustified non-adherence to treatment guidelines by healthcare professionals (West et al. 2015). The extent of medication wastage was studied and documented by various countries with quantities ranging from 65 unused medication items gathered from 73 households (Kiyingi &amp; Lauwo 1993) to 20,304 medications packages collected from 100 community pharmacies (Ekedahl et al. 2003). Medication wastage poses a financial burden on the nation&rsquo;s economy, as well as, environmental implications associated with its disposal. At present, Brunei Darussalam has a universal healthcare system run by the government with its services and funding provided, mainly, by the government. The citizens pay a very minimal amount for their medicines when treated in a government hospital or health centre and, thus, there would be financial implications to the government as a result of medication wastage. In 2009, the Department of Health in the United Kingdom explored the scale and cost of medication wastage, whereby, the direct cost of unused prescription medication to the National Health Services (NHS) amounted to &pound;300 million annually (Trueman et al. 2010).</div>\r\n<div>\r\n	In 2006, the Department of Pharmaceutical Services, Ministry of Health Brunei (MOH), has implemented the &lsquo;Return Your Medicines&rsquo; Programme. Through this programme, patients either from home or in the wards can return their unused medicines for safe disposal or recycling of medicines by MOH. The medicines being recycled will be evaluated and assessed using a set of protocol developed by the pharmacists from MOH. Many factors have contributed towards this excessive medicines storage, amongst them are patients died; non-adherence to their medications; change or cessation of treatment regime; and polypharmacy according to the Ministry of Health Malaysia (MOH 2013). An earlier study indicated that patients were afraid that their medications will be unavailable when they need them. Consequently, they tend to stock up on their medications (West et al. 2015).</div>\r\n<div>\r\n	Medication wastage also continues to compromise public health in terms of safety and the environment. The extent of the problem of unused medication in Brunei Darussalam was not yet known. This study aimed to investigate methods of disposing excess or unused medications and discussed the practices and attitudes of the participants towards unused medication.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This cross-sectional study was done at SSB hospital was designed to elicit information on the patients&rsquo; knowledge of attitudes towards and practices on the disposal of unused medicines, by means of a self-administered anonymous questionnaire survey. This study was conducted in the second largest public hospital in Brunei (183 beds). The population size (N) of patients at the outpatient pharmacy in the hospital was around 9,744 per month on average (SSB pharmacy 2012 statistics). The expected prevalence (p) of the study was 80%. Using the calculation formula by Naing et al. (2006), the sample size (n) was found to be 246 patients, who, were later selected using systematic sampling. In October 2013, over a period of two weeks, patients from outpatient pharmacy were asked to fill in the questionnaire to identify the following objectives; i) to find out the participants&rsquo; demography; ii) to identify the number of participants who were given advice on medicine disposal by medical/health practitioners; iii) to explore the participants&rsquo; knowledge and attitudes towards the methods of disposing unused medicines; v) to find out the participants&rsquo; preferred disposal practices of unused medicine; and vi) to discover the factors that caused them to keep the unused medications.</div>\r\n<div>\r\n	Written consent was attained from all the participants and permission was granted from the Director General of Medical Services in Ministry of Health to perform this study. Ethical approval was also granted by the Medical and Health Research Ethics Committee (MHREC) from the Ministry of Health, Brunei Darussalam. Patients could also refuse to participate (the number of participants who refused to take part was not recorded).</div>\r\n<div>\r\n	The questionnaire was designed to meet the study&rsquo;s objectives and was produced in Malay and in English depending on the preference of respondents. Similar studies were done in Serbia and New Zealand (Braund et al. 2009; Kusturica et al. 2012; Seehusen &amp; Edwards 2006) and their methods were considered and some parts of the questionnaire were utilised in this study. The author agreed for their questionnaire to be reproduced and used. The questionnaire consisted of three parts and thirteen questions altogether. The first part probed their socio-demographic characteristics (gender, age, education and occupation). The second part of the questionnaire focused on patients&rsquo; knowledge about medication disposal. The third part of the questionnaire queried the patients&rsquo; medication disposal attitudes and practices. This section focused on determining the factors, which cause them to stock unused medications. The survey questionnaire was adapted from Seehusen and Edwards (2006) and it was pilot tested for 20 participants and modified accordingly for validity.</div>\r\n<div>\r\n	The inclusion criteria were: i) patients aged 18 yrs and ii) who visited the outpatient pharmacy at SSB hospital during the study period. The study excluded all patients visiting the Extended Accident and Emergency Dispensary Services EA&amp;E, patients admitted to the hospital (i.e. inpatients) and those who could not write or read. All data were coded and entered into the IBM Statistical Package for Social Sciences Results IBM (SPSS Version 20) for statistical analysis. Chi-squared test was performed where appropriate and the level of significance was taken with a p value of less than 0.05 (p&lt;0.05).</div>\r\n<div>\r\n	RESULTS&nbsp;</div>\r\n<div>\r\n	The number of patients who came to the pharmacy waiting room, during the two-week data collection period, was around 3402. Out of the 3402 patients, 253 participants were systematically sampled and included for the study.</div>\r\n<div>\r\n	Factors contributing to keeping unused medicines at home</div>\r\n<div>\r\n	Figure 1 showed the frequency against the reason for keeping unused medicines at home, which, could lead to wastage. A total of 54.2% (n= 137) of the participants still kept their unused medicines because their medical condition had improved or had totally resolved. The second and third main reasons for having unused medicines were due to the side effects of the medication (n = 86, 34%) and due to the medicine which expired (n = 78, 30.8%). Only 68 patients (26.9%) mentioned that excess medicines were supplied to them. Patients who were deceased had the least amount of unused medicines at home (n = 24, 9.5%).</div>\r\n<div>\r\n	The extent of medication wastage and patient&rsquo;s demography</div>\r\n<div>\r\n	Almost equal number of men and women participated in this study. A total of 75.1% (n = 189) admitted to having unused medicines in their households. Majority of them were employed (n = 158, 62.5%). A total of 42.3% of them were educated at secondary level and 33.6% were educated at a minimum undergraduate level. It was noted that their ages were mainly within the working-age group population (18 yrs to 59 yrs). Only 3.6% (n= 9) of the participants were above 60 yrs. Almost half (47%, n = 119) of the participants admitted to keeping at least one to five medications in their households (Table 1). The types and costs of medications were not questioned in this study.</div>\r\n<div>\r\n	Participants&rsquo; knowledge on &ldquo;Return your medicine&rdquo; programme and disposal methods</div>\r\n<div>\r\n	Table 2 showed the participants&rsquo; responses on medicine disposal. A total of 67.6% of them had not been given proper advice on disposal of medication. Significantly, 50.2% (n = 127) of participants seemed to know that throwing medication in the household garbage, toilet or sink would have detrimental effects on the environment or/and public health. When participants were asked to rank the acceptable ways to deal with unused or expired medications, majority (70%, n = 177) of them agreed that garbage disposal was an acceptable method. This was followed by returning the medicines to the pharmacy, storing and keeping them within the household, throwing them into the sink/toilet, burning them off and lastly, by giving to their friends (Table 2).</div>\r\n<div>\r\n	Knowledge versus attitudes and practices of disposal of unused medicine</div>\r\n<div>\r\n	Table 3 showed the extent to which the participants&rsquo; knowledge has influenced them to return unused medication to the pharmacy (p= 0.065). Apparently, there was no significant association between knowing medicine effects on the environment and returning their unused medicine to the pharmacy. In other words, this knowledge did not influence participants to avoid throwing unused medicine in the garbage, sink or toilet.</div>\r\n<div>\r\n	In Table 2, over 29.2% (n = 74) of participants indicated that they had been given advice by a medical provider on ways to dispose expired or unused medication. Referring to Table 4, the prevalence (proportion) of practicing proper disposal methods between participants who had been given proper advice on disposal and those who have not was found to be insignificant for all variables (i.e. p values range from 0.305 to 0.798). Therefore, there was no significant association between getting a disposal advice by medical/healthcare providers and practicing &lsquo;acceptable&rsquo; disposal methods.</div>\r\n<div>\r\n	Table 5 compared the influence of educational background and their preferred method when disposing unused medicines. There was no significant association seen for all practices except the practice of burning (p =0.046). There was a significant association between education level and medicine disposal practice by burning.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	General findings</div>\r\n<div>\r\n	This study found that 75.1% of participants have kept their unused medicines at home and 65.5% kept between one to ten unused medicines. The rate was considered high since the study was only done within two weeks. The main factor was due to improvement in their medical condition. When asked about their knowledge on medicine disposal, 50.2% (n = 127) of participants seemed to know that throwing medication in the garbage, toilet or sink has a detrimental effect on the environment and/or public health. There was a significant association between educational background and the practice of burning of unused medicines.</div>\r\n<div>\r\n	Cost saving from medication recycling</div>\r\n<div>\r\n	Overall, 67.6% of the patients were not given proper advice by the medical/healthcare professionals on safe practices when disposing medication. If they were aware about the &ldquo;Return your medicines&rdquo; program, the unused medicines could have been returned back to the pharmacy and be recycled. Redistributing the unused medications to those who need them may potentially reduce non-compliance and the associated healthcare costs. There was a pilot study on the feasibility of medication recycling, following certain protocols, as a solution to medication wastage conducted in Singapore (Toh &amp; Chew 2016). This is a desirable practice and hospitals in Brunei have already practiced similar recycling through their in-patient pharmacies. In the current unpredicted economic situation, this practice is financially desirable, which, could lead to cost saving. In this study, however, the types of medicines being kept at homes and the actual cost of the medicines that can be recycled were not investigated.</div>\r\n<div>\r\n	Factors contributing to medicine wastages</div>\r\n<div>\r\n	A number of factors have been attributed to medication wastage. Our findings were similar to other studies conducted in this subject matter (Braund et al. 2009; James et al. 2009; Langley et al. 2005); 54.2% (n = 137) of participants still kept their unused medicines because their medical conditions have improved or have totally resolved. The second and third main reason for having unused medicines were due to the side effects of medicines (n = 86, 34%) and the medicine being expired (n = 78, 30.8%). In this study, patient death was the least likely reason as to why unused medicines were hoarded, perhaps this was because, most of the respondents were mainly patients in the working group category, and most elderly patients refused to participate. Only 6 participants were above 60 yrs. In a recent study (West et al. 2015), the most prevalent factor that caused medication wastage was that &ldquo;patients are afraid that their medication will be unavailable when they need it&rdquo; and therefore they overstocked. Fortunately, it was not the case in this study, as the Pharmacy in MOH Brunei have a mechanism to avoid this, such as, repeat dispensing within a short period of time to avoid wastages. Only 68 patients (26.9%) mentioned that excess medicines were supplied to them.</div>\r\n<div>\r\n	Practices of unused medicines disposal</div>\r\n<div>\r\n	Referring to Table 2, most participants 50.2% (n = 127) knew that throwing unused medications in the garbage, toilet or sink has detrimental effects on the environment or/and public health. When participants were asked to rank the acceptable ways to deal with unused or expired medications, majority (70%, n = 177) agreed that throwing the medicine in the garbage is an acceptable disposal method. This was followed by returning medication to the pharmacy, storing medication in the house, throwing in the sink, burning and lastly, they would least likely give them to their friends. Such findings were similar to other studies that have been conducted (Braund et al. 2009; Kusturica et al. 2012). There was no significant association found between getting an advice on disposal method and the knowledge of acceptable disposal methods of unused medicines. In fact, a significant association was found between those who got advice on proper disposal and those who practice disposal &lsquo;in the garbage&rsquo;, &lsquo;return to a pharmacy&rsquo; and &lsquo;storing medication in the house&rsquo;. The education level had not resulted in any significant association with disposal practices, except for those who had primary education, where they actually practiced burning more. However, burning remains the least practiced disposal method.</div>\r\n<div>\r\n	In conclusion, participants had poor knowledge on the proper disposal methods for unused medicines and this was also reported in other studies (Seehusen &amp; Edwards 2006). The USA study reported that less than 20% of the 301 participants received a medical provider advice on medication disposal compared to just less than 30% found in this study. Moreover, 35% of participants from the USA study said it was acceptable to flush medication down the toilet and 66.7% believed it was acceptable to return the unused medication to a pharmacy. In this study, 15.8% of the participants accepted flushing down the toilet whilst 47.4% preferred to return medication to the pharmacy. These figures indicate that the majority of SSB hospitals patients know that returning of unused medicines to the pharmacy is probably safer than disposing it themselves.</div>\r\n<div>\r\n	The attitudes and practices of the SSB hospital patients was that the majority (70%, n = 177) of the patients throw their unused medicines in the garbage. One study reported in Serbia (Kusturica et al. 2012) indicate a relatively high rate of throwing unused medication in the garbage (the total of urban and rural families was 80.3%, n = 159). Another study conducted in Kuwait (Abahussain et al. 2006) also reported 76.5% (n = 230) for the same disposal method. In Sweden, however, throwing medicines in the garbage was very low (3%) and instead, storage of unused medicines (55%) and returning unused medicines to the pharmacy (43%) were the two most acceptable practices (Persson et al. 2009).</div>\r\n<div>\r\n	What was also interesting was that despite knowing that the unused medicines had a negative impact on the environment, this did not influence patients to correctly dispose their unused medicines. Perhaps, being aware of the issue does not necessarily mean that the patients appreciate the impact and quantify the true risks. The consequence of continuing such attitudes and practices is the potential presence of pharmaceuticals in groundwater, lakes, rivers and even drinking water. In the long run, the impact on the environment is huge and this would continue to grow with the population growth and the increase in amount of dispensed medication.&nbsp;</div>\r\n<div>\r\n	When designing the survey, similar study done in Serbia (Seehusen &amp; Edwards 2006) was considered and adapted. The study sample was drawn from the patients of one hospital&rsquo;s outpatient pharmacy and the patients were mainly living in the Belait district (second largest district in Brunei). It was unclear how well these findings can be generalized for the entire Brunei population. Most of the elderly refused to participate in this study and the elderly were prone to have polypharmacy. This study did not explore the details of garbage disposal and whether the FDA standards were followed or not and there was no differentiation between the disposal of solid versus liquid medicines. It was highly likely that the liquid medicines were being flushed down sinks or toilets as reported in other studies (Braund et al. 2009). The exact type of medicines being kept at home and its relative costs were also not asked.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	This study indicates that patients from this outpatient pharmacy have poor knowledge and practices on the proper disposal of their unused medicines. Patient education on the &ldquo;Return your medicines&rdquo; program could possibly lead to them returning their unused medicines to the pharmacy. Various strategies could be made to reduce medication wastages. Health policy makers need to be aware of medication wastage and how preventing it can potentially reduce the cost of healthcare. Regarding the environmental effects due to poor practices of medicines disposal, education is again a key area to improve patients&rsquo; practices and beliefs. Policy guidelines on reducing medication wastages and saving the environment need to be promoted. Future studies in this area can be done in a larger public hospital to identify the types of unused medication and their relative costs.</div>\r\n<div>\r\n	ACKNOWLEDGEMENT</div>\r\n<div>\r\n	The authors would like to acknowledge the pharmacist who granted permission for the study to be conducted within their premises and also to all participants in this study. The authors also acknowledge Seehusen and Edwards (2006), who had permitted us to use few parts of their survey questions.</div>\r\n', 1);
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(67618, 67604, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Neurodegenerative diseases are diseases caused by the loss of neuronal function in the brain. If the loss due to cell damage, the brain region cannot function properly and this will result in reduced brain volume. The contributing risk factors include both genetic and environmental impacts. In particular, environmental factors such as toxic exposure, stress and unhealthy food were recognized to play a crucial role in modifying the risk of neurodegenerative diseases. Several known neurodegenerative diseases are Alzheimer&rsquo;s, Parkinson&rsquo;s, encephalitis, epilepsy, genetic brain disorders, stroke, multiple sclerosis, Huntington&rsquo;s and depression (Prilipko et al. 2004). Moreover, oxidative stress induced neuronal cell damage was reported to be involved in neurodegenerative diseases such as Alzheimer&rsquo;s disease and depression (Kim et al. 2015; Xie &amp; Chen 2016). The damage is mediated by the accumulation of free radicals include reactive oxygen species (ROS), mainly superoxide anion (O2-) and hydrogen peroxide (H2O2) in cells. However, several studies showed that antioxidants can inhibit ROS (Andersen 2004; Fatokun et al. 2008).</div>\r\n<div>\r\n	Presently, there are no ideal drugs for neurodegenerative diseases. The desired drugs should possess high efficacy, no side effects and low cost. Development of preventive and therapeutic approaches for neurodegenerative diseases should be ongoing. According to previous studies, rice and corn, our fundamental foods, are beneficial, not only as our main carbohydrate source, but also preventive and therapeutic effect with respect to neurodegenerative effects (Torre et al. 2008; Ismail et al. 2012). This might be due to the chemical known as melatonin (N-acetyl-5-methoxytryptamine) found in rice and corn since melatonin can function as an antioxidant (Mamiya et al. 2007). Melatonin was also shown to help reducing sleeplessness in depressive patients (Lewy et al. 1998; Trotti &amp; Karroum 2016). Furthermore, it was demonstrated to exhibit the favorable effects for patients with Alzheimer&rsquo;s (Ng et al. 2010) and Parkinson&rsquo;s diseases (Miller et al. 1996; Breen &amp; Barker 2016).&nbsp;</div>\r\n<div>\r\n	Melatonin is a chemical compound derived from the metabolism of L-tryptophan (Cardinali &amp; P&eacute;vet 1998). Melatonin is not only found in mammals, but also in various herbs (Hattori et al. 1995; Dubbels et al. 1995). Rice and corn are considered to be the main agricutural product cultivated in Thailand, and they are reported to contain melatonin (Hernandez-Ruiz &amp; Arnao 2008). Regarding its molecular meachanism of action, several lines of evidence showed that melatonin exterted its effects by upregulating the expression of brain-derived neurotrophic factor (BDNF) gene (Imbesi &amp; Manev 2008; Imbesi et al. 2008; Zhang et al. 2013). This particular molecule mediates its effects by enhancing both the function of the nerve cells and the anti-aging activity of the brain cells via kinase signaling pathway (Mizuno et al. 2003). In the present study, we examined the effect of white rice, brown rice, black glutinous rice, sweet corn and baby corn extracts against H2O2-induced neurotoxicity and their underlying mechanisms in the mouse hippocampal HT22 cells.</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	Chemicals</div>\r\n<div>\r\n	Chemicals such as 2,2&#39;-azino-bis-3-ethylbenzothiazoline-6-sulfonic acid (ABTS), 2,2-diphenyl-1-picrylhydrazyl (DPPH), potassium persulfate, quercetin, Dulbecco&rsquo;s modified Eagle medium (DMEM) and fetal bovine serum (FBS) were purchased from Sigma-Aldrich Co. (St Louis, MO, USA). Other chemicals such as 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) were purchased from Bio basic (Toronto, Canada). Ascorbic acid (vitamin C), dimethyl sulfoxide (DMSO) and Hydrogenperoxide (H2O2) were purchased from Merck (Darmstadt, Germany). Methanol, ethanol, hexane were purchased from RCI Labscan (Bangkok, Thailand). Penicillin-streptomycin solution was obtained from Corning Inc. (Corning, NY, USA). Annexin V FITC and PI kit were purchased from Biolegend (San Diego, CA, USA) and H2DCF-DA was purchased from Life technology (Carlsbad, CA, USA).</div>\r\n<div>\r\n	Cell culture</div>\r\n<div>\r\n	The HT22 cells (a generous gift from Prof. Dr. David Schubert at the Salk Institute, San Diego, CA, USA) were cultured in DMEM supplemented with 10% heat inactivated FBS and 1% penicillin/streptomycin in a 5% CO2 humidified incubator at 37˚C. The cells were passaged by trypsinization once they reached approximately 80% confluence.</div>\r\n<div>\r\n	Collection of rice and corn samples</div>\r\n<div>\r\n	Five types of Thai plants (rice and corn samples) in this study were collected from the market at Chiangrai, the Northern Province, Thailand. All of them were identified and deposited at the Prof. Dr. Kasin Suvatabhandhu Herbarium, Department of Botany, Faculty of Science, Chulalongkorn University, Thailand. Their details such as scientific names, parts used and herbarium numbers for this study were presented in Table 1.</div>\r\n<div>\r\n	Preparation of rice and corn extracts&nbsp;</div>\r\n<div>\r\n	All glutinous rice and corns were ground with a mortar and pestle. Ground samples were add with solvent water, ethanol and hexane (ratio 1:10). Samples with water were extracted by heating up. Briefly, each sample was mixed with distilled water and then kept in water bath at 70oC for 30 mins. The supernatants were collected, passed through Whattman no. 1 filter paper, and then dried using Moduly OD freeze Dryer. For samples extracted using ethanol and hexane, they were extracted by maceration. Briefly, samples were shaking in the incubator shaker at 25 oC and 48 hrs. The supernatant was filtered through Whattman no. 1 filter paper, and then pooled and dried using rotatory evaporator to concentrate the extract. Finally, all of the plant extracts were dissolved in DMSO in the concentration of 100 mg/mL as stocks, and stored with protection from light at -20 oC until use.</div>\r\n<div>\r\n	Measurement of cell viability using MTT assay&nbsp;</div>\r\n<div>\r\n	To determine the ability of rice and corn extracts to protect HT22 cells from H2O2, MTT assay was performed. Briefly, HT22 cells were seeded into 96-well culture plates at density of 1 x 104 cells/mL and were allowed to attach. After 24 hrs, the cells were treated with rice and corn extracts individually in a concentration range of 1.56&ndash;100 &mu;g/mL for 24 hrs. Similarly, cells were treated with H2O2 in a concentration range of 31&ndash;2000 &mu;M up to 24 hrs. For the determination of neuroprotective effect, cells were pretreated each rice and corn extract diluted in medium for 24 hrs and then challenged with H2O2 for another 24 hrs. Rice and corn extracts were dissolved in DMSO which was maintained at 0.1% as this concentration showed no toxicity to the cells. MTT was added to all wells and allowed to incubate in dark at 37˚C for 4 hrs. The amount of MTT formazan product was determined by measuring absorbance at 550 nm using a microplate reader. The results were expressed as % cell survival, assuming as 100% the absorbance in control untreated cells. All the MTT assays were performed in triplicate.</div>\r\n<div>\r\n	Assessment of apoptosis using flow cytometry</div>\r\n<div>\r\n	To detect the effects of rice and corn extracts on number of apoptotic cells induced by H2O2. The HT22 cells were stained with FITC-conjugated Annexin V and propidium iodide. Briefly, HT22 cells were seeded in 6-well plates at density of 2 x 105 cells/mL. The cells were pretreated with rice and corn extracts with water, ethanol and hexane at concentration 100 &mu;g/mL for 24 hrs before being exposed to 250 &mu;M H2O2 for another 24 hrs. The cells were trypsinized and cell pellets were resuspended in ice-cold 1 x binding buffer. Annexin V-FITC solution 5 &mu;L and propidium iodide 10 &mu;L were added to 100 &mu;L of cells suspension. The tube was incubated on ice for 15 mins in the dark followed by addition of 400 &mu;L ice-cold 1 x binding buffer and mixing gently. The samples were analyzed using flow cytometer (FACS Calibur, BD Biosciences, San Jose, CA, USA).</div>\r\n<div>\r\n	Measurement of free radical scavenging using 1,1-diphenyl-2-picrylhydrazyl (DPPH assay)</div>\r\n<div>\r\n	To investigate the antioxidant property of rice and corn extracts. &nbsp;Generating the stable free radical DPPH (DPPH&bull;), DPPH was dissolved in absolute ethanol. DPPH solution was prepared daily for fresh, and absorbance of the DPPH solution was measured at 517 nm. Then, 20 &mu;L of the extracts (1 mg/mL) or Ascorbic acid (serving as a standard) (125 &ndash; 1 &mu;g/mL) was mixed with 180 &mu;L of DPPH solution and incubated for 30 mins in the dark at room temperature. The absorbance of the reaction mixture was measured at a wavelength of 517 nm. The results are expressed as mg ascorbic acid equivalent per g fresh weight of sample. The % scavenging activity (% SC) was calculated using the following formula:&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	% SC = {[Abs. control - (Abs. sample - Abs. blank sample)]/Abs. control} * 100</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Control included 180 &mu;L of DPPH solution and 20 &mu;L of absolute ethanol; whereas, blank sample included 180 &mu;L of absolute ethanol and 20 &mu;L of extracts.</div>\r\n<div>\r\n	Measurement of free radical scavenging using 2,2-azinobis (3-ethylbenzothiazoline-6-sulfonic acid) radical (ABTS assay)&nbsp;</div>\r\n<div>\r\n	To investigate the antioxidant property of rice and corn extracts. The working solution or ABTS&bull;+ solution was freshly prepared by mixing 7 mM ABTS stock solution and 2.45 mM K2S2O8 at ratio 8:12, and was subsequently incubated for 16&ndash;18 hrs at room temperature in the dark. Thereafter, ABTS&bull;+ solution was diluted by mixing with absolute ethanol at ratio about 1:20 to obtain an absorbance of 0.700 &plusmn; 0.020 at 734 nm. Then, 20 &mu;L of the extracts (1 mg/mL) or ascorbic acid (serving as a standard) (60&ndash;1 &mu;g/mL) was mixed with 180 &mu;L of ABTS&bull;+ solution for 45 mins in the dark. The absorbance was measured at a wavelength of 734 nm. The results are expressed as mg ascorbic acid (vitamin C) equivalent per g fresh weight of sample. The % SC was calculated using the following formula:&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	% SC = {[Abs. control - (Abs. sample - Abs. blank sample)]/Abs. control} * 100</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Control included 180 &mu;L of ABTS&bull;+ solution and 20 &mu;L of absolute ethanol; whereas, blank sample included 180 &mu;L of absolute ethanol and 20 &mu;L of extracts.</div>\r\n<div>\r\n	Determination of Tryptophan and Melatonin content in rice and corn extracts by HPLC</div>\r\n<div>\r\n	To determine tryptophan and melatonin content in rice and corn extracts. A 20 &mu;L samples were injected into an Eclipse XDB C18 column (15 cm x 45; 5 &mu;m) No. 1. A gradient elution program was used with two mobile phases: A (1% acetic acid in water) and B (absolute methanol). The applied gradient was as follows: (time, solvent B): 0.1 mins, 10% ; 15 mins, 40% ; 20-30 mins, 70% ; 32-50 mins, 10%. The flow rate of tryptophan and melatonin were fixed at 1 mL/min. The contents of tryptophan and melatonin were detected using DAD UV detector at 280 nm.</div>\r\n<div>\r\n	Measurement of reactive oxygen species (ROS) using flow cytometry&nbsp;</div>\r\n<div>\r\n	To determine the level of intracellular ROS, HT22 cells were seeded in 6-well plates at density of 2 x 105 cells/mL. The cells were pretreated with black glutinous rice extract with ethanol, baby corn extract with water and baby corn extract with ethanol 100 &mu;g/mL for 48 hrs before being exposed to 250 &mu;M H2O2 and H2DCF-DA for another 30 mins at 37˚C under 5% CO2. The cells were subsequently harvested and washed with phosphate-buffered saline (PBS). Fluorescence intensity was measured using flow cytometry (FACSCalibur, BD Biosciences, San Jose, CA, USA), with an excitation wavelength of 488 nm and an emission wavelength of 525 nm.&nbsp;</div>\r\n<div>\r\n	Measurement of BDNF mRNA expression by Real-time PCR</div>\r\n<div>\r\n	To investigate the mRNA level expression of BDNF mRNA, total RNA was isolated with Trizol (Invitrogen, Carlsbad,CA). An amount of 1 &mu;g of RNA were reverse transcribed using Super-Script&trade; II Reverse Transcriptase (Invitrogen) and 0.5 &mu;g OligodT (Bio basic, Toronto, Canada) in a 20 &mu;l final volume. Real-time PCR was carried out using the Exicycler Real Time Quantitative Thermal Block (Bioneer, Daedeok-gu, Korea) and SYBR Green was used for the detection of double-strand DNA. The PCR reaction was set up into microtubes in a volume of 25 &mu;l with 1 &mu;l of cDNA and 24 &mu;l of Master mix SYBR Green I (Bioneer). For BDNF quantitation, fwd AACCATAAGGACGCGGACTTG and rev TTGACTGCTGAGCATCACCC primers were used, while for &beta;-actin quantitation, used as internal control, fwd GGCTGTATTCCCCTCCATCG and rev CCAGTTGGTAACAATGCCATGT primers were used. The PCR program included an initial denaturation step during 10 mins at 95˚C, followed by 35 cycles of a 15 s melting step at 95˚C, a 15 s annealing step at 57˚C, and a 30 s elongation step at 72˚C. At the end of each cycle, the fluorescence emitted by SYBR Green was measured. At the end of PCR reaction, samples were subjected to a temperature ramp (from 60˚C to 94˚C, 1˚C/s) with continuous fluorescence monitoring for melting curve analysis. For each PCR product, a single narrow peak was obtained by melting curve analysis at the specific temperature. The analysis was performed with Light Cycler Relative Quantification Software. Samples containing no template were used as negative controls in each experiment.</div>\r\n<div>\r\n	Measurement of BDNF protein expression by Western blot analysis&nbsp;</div>\r\n<div>\r\n	To investigate the protein level expression of BDNF protein, HT22 cells were seeded in 6-well plates at density of 2 x 105 cells/mL. The cells were pretreated with black glutinous rice extract with ethanol, baby corn extract with water &nbsp;and baby corn extract with ethanol 100 &mu;g/mL for 24 hrs before being exposed to 250 &mu;M H2O2 for another 24 hrs. The cells were lysed with lysis buffer (50 mM Tris [pH8.0], 150mM NaCl, 1% NP40, 1mM PMSF, 1mM DTT). The total protein concentrations of the lysates were determined using the Bradford protein assay and &nbsp;proteins (20 &mu;g) were loaded on 12% Sodium dodecyl sulfate polyacrylamide gel electrophoresis gels (SDS-PAGE gels), transferred onto polyvinylidene fluoride (PVDF) membranes (Biorad, Hercules, CA, USA) and incubated overnight with primary and 1 hr with appropriate secondary antibodies. Primary antibodies were diluted as follows: rabbit anti-&beta; actin (Cell signalling, Danvers, Massachusetts, USA, #4967) 1:16,000; rabbit anti-BDNF (Abcam, Cambridge, UK, ab72439) 1:1,000. Secondary horseradish peroxidase-conjugated antibodies (Cell signalling, Danvers, Massachusetts, USA, #7074) were diluted 1:16,000. The expression of &beta;-actin, a housekeeping gene, was used for normalization. Western blotting data were reproduced three times independently. Intensity of the bands was estimated using the Quantity One software (Biorad).</div>\r\n<div>\r\n	Statistical analysis&nbsp;</div>\r\n<div>\r\n	All experiments were performed independently at least three times and in triplicates or quadruplicates as indicated. Data was expressed as the mean &plusmn;&plusmn;SE of the mean (SEM). The statistical probability for correlation coefficients was calculated using Statistical Package for Social Science (SPSS) version 17 (IBM Corporation, Armonk, NY, USA). Statistical analysis was conducted by one-way ANOVA and Student&rsquo;s t-test. &nbsp;P &lt; 0.05 was considered as significant.&nbsp;</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Extraction yield of rice and corn extracts</div>\r\n<div>\r\n	The percentage yield of all five Thai plants that were successively extracted with water, ethanol and hexane ranged from 0.17% to 18.23%. Furthermore, the highest percentage yields were obtained from water fractions in Table 2.</div>\r\n<div>\r\n	Effect of Rice and Corn extracts on cell viability induced by H2O2 in HT-22 cells</div>\r\n<div>\r\n	We investigated the effects of white rice, brown rice, black rice, sweet corn and baby corn on H2O2-induced cell death in HT-22 cells. HT-22 cells exposed to H2O2 in the absence and presence of pretreated with white rice, brown rice, black rice, sweet corn and baby corn was evaluated using MTT assay. The results showed that exposure of HT-22 cells to white rice, brown rice, black rice, sweet corn and baby corn extract with water, ethanol and hexane separately up to 24 hrs over concentration range of 1.56-100 &micro;g/mL produced no alteration in cell viability as compared to untreated control. So, all of these plant extracts showed no toxicity to HT-22 cells (Figure 1A), (Figure 1B) and (Figure 1C).</div>\r\n<div>\r\n	On the other hand, exposure of cells to 250 &mu;M H2O2 for 24 hrs resulted in approximately 60% cell cytotoxicity in comparison to control cells (Figure 2). Therefore, 250 &mu;M H2O2 was chosen for incubation of HT-22 cells for 24 hrs to induced cell death in all subsequent experiments.</div>\r\n<div>\r\n	In contrast, pre-treatment with Rice and Corn extract with water, ethanol and hexane after that exposure to 250 &mu;M H2O2, the result showed that the protective effect of white rice, brown rice and black rice extract with water were no significant, but baby corn extracts with water at 25, 50, 100 &mu;g/mL and sweet corn extracts with water at 100 &mu;g/mL were significantly (p &lt; 0.05) increased the viability of HT-22 cells against H2O2-induced cytotoxicity (Figure 3A). In addition, exposure of HT-22 cells to white rice extracts with ethanol were no significant, while brown rice and black rice extract with ethanol at concentration 50 and 100 &mu;g/mL, sweet corn and baby corn at concentration 100 &mu;g/mL were significantly (p &lt; 0.05) increased the viability of HT-22 cells against H2O2-induced cytotoxicity (Figure 3B). Furthermore, exposure of HT-22 cells to white rice extracts with hexane were no significant, however sweet corn and baby corn extract with hexane at concentration 25, 50 and 100 &mu;g/mL and brown rice extracts with hexane at concentration 50, 100 &mu;g/mL and black rice extracts with hexane at concentration 100 &mu;g/mL were significantly (p &lt; 0.05) increased the viability of HT-22 cells against H2O2-induced cytotoxicity. The result show that brown rice, black rice, sweet corn and baby corn extract with hexane (Figure 3C) were able to neutralize the effect of 250 &mu;M H2O2. Quercetin (10 &mu;M) was used as sample control.</div>\r\n<div>\r\n	Effect of Rice and Corn extracts on H2O2 induced apoptosis in HT22 cells</div>\r\n<div>\r\n	In this study, we evaluated the protective effect of rice and corn extracts on apoptosis of the HT-22 cells using flow cytometry with Annexin V-FITC/PI double staining. The H2O2-treated cells (Pos) significantly increased the number of the apoptotic cells (Figure 4A,4B). The percentages of the apoptotic cells at concentration 100 &mu;g/mL of pretreatment with brown rice extracts with hexane (BrHe), black rice extracts with ethanol (BlE) and hexane (BlHe), baby corn extracts with water (BaH) and ethanol (BaE), sweet corn extracts with water (SH) and hexane (SHe) were 48.2%, 31.27%, 35.66%, 42.52%, 49.37%, 47.67% and 43.325% respectively and were found to be significantly (p &lt; 0.05) lower when compared to the percentages of the cells treated with H2O2 alone at 51.47% &nbsp;(Figure 4A, 4B). While, brown rice extracts with ethanol (BrE), baby corn extracts with hexane (BaHe) and sweet corn extracts with ethanol (SE) were not significant. These results indicate that brown rice extracts with hexane (BrHe), black rice extracts with ethanol (BlE) and hexane (BlHe), baby corn extracts with water (BaH) and ethanol (BaE), sweet corn extracts with water (SH) and hexane (SHe) prevented neuronal cell apoptosis.&nbsp;</div>\r\n<div>\r\n	Antioxidant activity of Rice and Corn extracts</div>\r\n<div>\r\n	To determine the antioxidant activities, DPPH and ABTS assay were used. DPPH assay is based on hydrogen donor property of antioxidants and is widely used in natural antioxidant studies because of its sensitivity and simplicity. ABTS assay has also widely used to evaluate antioxidant activities caused its can detect in both aqueous and lipid phase. The result of the DPPH and ABTS assay are listed in Table 3 and 4, respectively. In both of antioxidant activity assays, black rice extract from ethanol fraction had the richest antioxidant activity, DPPH assay (% Scavenging 13.44 &plusmn; 1.99 and mg Vit C g-1 fresh weight of sample 9.54 &plusmn; 1.21) and ABTS assay (% Scavenging 26.73 &plusmn; 0.42 and mg Vit C g-1 fresh weight of sample 17.80 &plusmn; 0.65). In addition, ABTS assay showed more extracts different DPPH assay including baby corn extracts with water and ethanol. &nbsp;Baby corn extracts with water had % Scavenging 23.42 &plusmn; 0.10 and mg Vit C g-1 fresh weight of sample 15.73 &plusmn; 0.35 and baby corn extracts with ethanol had % Scavenging 27.63 &plusmn; 0.93 and mg Vit C g-1 fresh weight of sample 18.36 &plusmn; 0.42 in Table 4.</div>\r\n<div>\r\n	Tryptophan and Melatonin content in rice and corn extracts</div>\r\n<div>\r\n	Tryptophan and Melatonin content in rice and corn extracts were determined by HPLC. Data of the tryptophan and melatonin level in rice and corn extracts are listed in Table 5. Baby corn extracts from water and ethanol fractions had the highest tryptophan contents at 2898.44 and 1303.09 &mu;g/kg, respectively. In addition, black rice extracts from ethanol fraction had the highest level of melatonin content at 396.38 &mu;g/kg. &nbsp;&nbsp;</div>\r\n<div>\r\n	Effect of Rice and Corn extracts on H2O2 induced production of reactive oxygen species (ROS) in HT22 cells</div>\r\n<div>\r\n	To investigate the protective effect of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) against &nbsp;H2O2 in the production of ROS, the cells were &nbsp;pretreated with 100 &mu;g/mL of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) for 24 hrs before being treated with 250 &mu;M H2O2 together with DCFDA for 30 mins. The results show that treated with H2O2 alone significantly (p &lt; 0.05) increased the level of ROS by approximately 90.4%, in comparison with the control group. Pretreatment with 100 &mu;g/mL of black glutinous rice extracts with ethanol (BlE) and baby corn extracts with ethanol (BaE) significantly reduced the level of the ROS production. In contrast, baby corn extracts with water (BaH) was no significant (Figure 5A,5B).</div>\r\n<div>\r\n	Effect of Rice and Corn extracts on H2O2 induced expression of BDNF mRNA in HT22 cells</div>\r\n<div>\r\n	To determine the protective effect of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) on the mRNA level expression of BDNF mRNA. The gene was monitored by quantitative real-time RT-PCR assay. Cell treated with 250 &mu;M H2O2 alone resulted in decreased expression of the BDNF genes expression. Pretreated with 100 &mu;g/mL of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) for 24 hrs before being treated with 250 &mu;M H2O2 the result show that (BlE), (BaH) and (BaE) were significantly (p &lt; 0.05) increased in BDNF expression levels 1.37, 1.52 and 2.00-folds respectively (Figure 6).</div>\r\n<div>\r\n	Effect of Rice and Corn extracts on H2O2 induced expression of BDNF protein in HT22 cells&nbsp;</div>\r\n<div>\r\n	To determine the protective effect of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) on the expression of BDNF protein. Cell treated with 250 &mu;M H2O2 alone resulted in decreased expression of the BDNF protein. Pretreated with 100 &mu;g/mL of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) for 24 hrs before being treated with 250 &mu;M H2O2 the result show that (BlE), (BaH) and (BaE) were significantly (p &lt; 0.05) increased in BDNF expression levels 1.08, 1.11 and 1.40-folds respectively, as compared to H2O2 group (Figure 7).</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The H2O2-induced cell damage has been shown to be involved in neurodegenerative diseases (Kim et al. 2015; Xie &amp; Chen 2016). Several studies have demonstrated the H2O2-induced neurotoxicity since it can directly induce apoptosis in neuronal cell by several mechanisms such as oxidative stress (Rao et al. 2013; Zhao et al. 2013). The present study demonstrated that rice and corn extracts may exhibit the neuroprotective effect since they contain melatonin and the precursor for its biosynthesis (tryptophan) and antioxidants. Also, the induced expression of BDNF level by our extracts demonstrated in this work is of great interest since this protein is believed to play a functional role against neurodegenerative diseases. Rice and corn were revealed to have the beneficial effects to the HT22 cells, and this might be due to the presence of such antioxidants as tryptophan and melatonin (Marshall et al. 1996; Pandi-Perumal et al. 2005).&nbsp;</div>\r\n<div>\r\n	As we first utilized the MTT assay for determining the neuroprotective of rice and corn extracts against H2O2 toxicity in cultured HT-22 cells, our current results demonstrated that rice and corn extracts significantly improved the cell viability after H2O2 exposure. In contrast, white rice extracts could not improve the cell viability after the presence of H2O2. To find the related mechanisms, we evaluated the protective effects of rice and corn extract against H2O2-induced neuronal apoptosis. The number of apoptotic cells was quantitatively analyzed using flow cytometry. We found that rice and corn extracts significantly protected the HT-22 cells from H2O2-induced cytotoxicity through the apoptosis pathway. Nevertheless, brown rice and sweet corn extracted using ethanol and baby corn extracted using hexane did not significantly protect the HT-22 cells from H2O2-induced cytotoxicity through the apoptosis pathway.&nbsp;</div>\r\n<div>\r\n	Numerous studies have demonstrated the antioxidant activities of rice and corn (Torre et al. 2008; Ismail et al. 2012; Soi-ampornkul et al. 2012; Thummayot et al. 2014). Our study actually demonstrated that our local rice and corn extracts possess varying degrees of antioxidant activities, depending on the extraction solvent types. We demonstrated that black rice extracts with ethanol, baby corn extracts with water and ethanol were shown to possess the highest content of antioxidants. We also determined the levels of melatonin and tryptophan in rice and corn extracts. Previously, there was a study revealing the level of melatonin content in black rice at 140 &mu;g/kg (Manchester et al. 2000; Hernandez-Ruiz &amp; Arnao 2008; Setyaningsih et al. 2014). Our results demonstrated that the highest levels of melatonin content were detected in black rice extracts from ethanol fraction at 396.38 &mu;g/kg. In addition, the highest levels of tryptophan content were detected in baby corn extracts from water and ethanol. These data demonstrated the contents of melatonin and tryptophan in line with the results of antioxidant activities.&nbsp;</div>\r\n<div>\r\n	We selected the best three fractions for further analyses, black rice extracts from ethanol fraction, baby corn extracts from water and baby corn extracts from ethanol fractions. Numerous studies, both in vitro and in vivo, revealed that H2O2 treatment significantly increased ROS production, which resulted in apoptosis (Andersen 2004; Fatokun et al. 2008; Rao et al. 2013; Zhao et al. 2013; Kim et al. 2015; Xie &amp; Chen 2016). Moreover, consistent with the findings from previous studies, we found that the levels of intracellular ROS had markedly increased after the treatment of H2O2 at 250 &mu;M. Pretreating cells with black rice extracts from ethanol fraction and baby corn extracts from ethanol fraction significantly suppressed H2O2-induced ROS accumulation, thereby suggesting that the antioxidant activity of black rice extracts from ethanol fraction and baby corn extracts from ethanol fraction may be useful for attenuating and preventing apoptosis in neurodegenerative disease (Ismail et al. 2012; Thummayot et al. 2014).&nbsp;</div>\r\n<div>\r\n	We finally explored the effect of black rice extracts from ethanol fraction, baby corn extracts from water and baby corn extracts from ethanol fraction on the expression of BDNF mRNA and protein in H2O2-induced HT-22 cells using real time PCR and Western blot. BDNF, recognized as a member of the &ldquo;neurotrophin&rdquo; family of growth factors, is a secreted protein encoded by the BDNF gene (Binder &amp; Scharfman 2004). Several studies have demonstrated that BDNF has a mechanism related to H2O2 toxicity. Cells exposed to H2O2 resulted in the decreased BDNF level (Huang &amp; McNamara 2012; Ghaffari et al. 2014). Moreover, several lines of evidence showed the preventive effect of BDNF on neurodegenerative diseases. In depression and Alzheimer&rsquo;s disease, when the hippocampus was also damaged, the reduced levels of the neurotrophic factor were documented. &nbsp;Therefore, antidepressants were applied to raise the levels of BDNF to protect and increase the volume of hippocampal and other cells (Mattson 2008). In the present study, we found that H2O2 is significantly decreased BDNF mRNA and protein levels in line with the previous findings. Interestingly, we found that pretreating HT22 cells with black rice extracts from ethanol fraction, baby corn extracts from water and baby corn extracts from ethanol fraction increased BDNF mRNA and protein levels. Pretreated cells with black rice extracts from ethanol fraction, baby corn extracts from water and baby corn extracts from ethanol fraction reversed this H2O2- induced neurotoxicity, suggesting the beneficial effects of rice and corn in vitro.</div>\r\n<div>\r\n	Our limitations for this investigation included plant samples collected one occasion only, cell-based assay (not systemic approach), plant extractions using few solvent types and limited and only BDNF gene determined. However, it was the first study demonstrating the advantageous effects of rice and corn extracts against H2O2-induced neurotoxicity, particularly the impact on BDNF expression in HT22 cells, thus highlighting their nutraceutical properties and health benefits.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Rice and corn including black rice extracts from ethanol fraction, baby corn extracts from water and baby corn extracts from ethanol fraction protected the neurotoxicity of HT-22 cells from H2O2. Inhibition of ROS production in conjunction with modulation of BDNF expression, by their endogenous contents of melatonin and tryptophan, may contribute to the underlying mechanisms.</div>\r\n<div>\r\n	Acknowledgments</div>\r\n<div>\r\n	This research was financially supported by the Korea Foundation for Advanced Studies (KFAS), Asia Research Center of Chulalongkorn University and the 90th anniversary (Ratchadaphiseksomphot Fund) of Chulalongkorn University Fund (Grant No. GCUGR11255725102M). The authors thank Prof. Dr. David Schubert for proving the HT22 cells for this study. SC was a recipient of the tuition fee scholarship provided by Faculty of Allied Health Sciences, Chulalongkorn University.&nbsp;</div>\r\n', 1),
(67628, 67614, '<div>\r\n	Introduction &nbsp;</div>\r\n<div>\r\n	Menopause is the permanent cessation of menstruation resulting from the loss of ovarian follicular activity which occurs after 12 consecutive months of amenorrhea without any pathological or physiological cause (WHO, 1994). The timing of natural menopause is variable, however, it can occur in the women with age range from 45-55 years. Menopause has detrimental effects on cardiovascular function and metabolism in women (Leong et al. 2008). Menopause is associated with cardiovascular disease (CVD) including hypertension, obesity, stroke, myocardial infarction and renal failure (Bray 2003; &nbsp;Leong et al. 2015). Among all the complications related to the menopause, obesity is an important one to be highligted. Postmenopausal obesity results in increased risk of many diseases, such as diabetes, hypertension, heart diseases, osteoporosis and breast cancer &nbsp;(Bray 2003). Several experimental studies used ovariectomised rats as a menopausal model for the tentative researches (Leong et al. 2015).&nbsp;</div>\r\n<div>\r\n	High Blood Pressure is defined as systolic blood pressure (SBP) greater than 140 mmHg and/or diastolic blood pressure (DBP) greater than 90 mmHg (Bray 2003). Increase in the blood pressure is the most common symptom in the CVD and is a main source of morbidity and mortality worldwide. Approximately, 90-95% cases of hypertension are idiopathic. It is a complex, multifactorial disorder, including genetic and lifestyle modifications (Leong et al. 2015). Based on the epidemiological surveys, several factors are linked with an increase in the blood pressure and these include age, sex, race, socioeconomic status, emotional disturbances, nutrition, increase in the body weight, alcohol consumption, physical activity and exposure to various environmental agents. In Malaysia, data from the National Health and Morbidity Survey &nbsp;reported that 37% of the population developed hypertension (Institute of Public Health 2008).&nbsp;</div>\r\n<div>\r\n	The occurrence of increase in blood pressure and CVD are less in women compared to men of similar age and this is due to the sufficient oestrogen level present in the reproductive age of the women (Zago et al. 2004). However, the oestrogen levels gradually diminish at the age of menopause and predispose to several diseases including obesity and hypertension. Oestrogen has been proven to exhibit cardioprotective effects in pre-menopausal women. A number of studies reported that the incidence of hypertension and menopausal are highly correlated (Zago et al. 2004; Deroo &amp; Korach 2006; Adam et al. 2009). Few studies presumed that the consumption of improper diet (high sodium diets, high cholesterol diet) has a high risk of developing elevated blood pressure in the postmenopausal state.&nbsp;</div>\r\n<div>\r\n	There are in vivo and in vitro studies which were conducted to observe the effect of cholesterol rich diet on major blood vessels (Jaarin et al. 2015; Nurul-Iman et al. 2013; Leong et al. 2015). Significant changes were observed in the pathophysiology of the aorta following the cholesterol rich diet in ovariectomised rats. It is noteworthy that the commercial cholesterol rich diet is cost expensive. Prolonged consumption of cholesterol diet (more than 24 weeks) led to develop hypertension in the ovariectomised rats (Jaarin et al. 2015; Leong et al. 2015). It was important to observe the effect of short term consumption of cholesterol diet on hypertension which may result in low consuming of cholesterol diet. The effect of 2% cholesterol diet on the blood pressure was also to be investigated. &nbsp;Hence, the present study was aimed to develop hypertensive ovariectomised rat model following the consumption of 2% cholesterol in the normal diet in a short term period.&nbsp;</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	Animals and diets&nbsp;</div>\r\n<div>\r\n	Eighteen female Sprague-Dawley rats with weighing 250 &plusmn; 50 g were obtained from Animal unit, Universiti Kebangsaan Malaysia. The rats were kept in stainless steel cage at temperature 20&ndash;22˚C and 12:12 h light cycle). The rats were acclimatized for 7-days and were given free access to food and water. Prior ethical approval was obtained. Two percent cholesterol diet was purchased from Next Gene Scientific Sdn Bhd, Singapore.</div>\r\n<div>\r\n	Study Design</div>\r\n<div>\r\n	The rats were randomly divided into three groups of six animals (n=6) each; sham control (Sham), ovariectomised control fed with standard rat chow (Ovx+N) and &nbsp;ovariectomised control fed with 2% cholesterol (Ovx+Cho). Following the ovariectomy, the treatment was continued for 4 weeks.</div>\r\n<div>\r\n	Procedure for ovariectomy</div>\r\n<div>\r\n	All the animals except Sham group were ovariectomised following intraperitoneal injection of ketamine hydrochloride and xylazine (50 and 10 mg/kg body weight, respectively). Bilateral ovariectomies were performed from a dorsal approach. Following 2 weeks of ovariectomy, Sham and Ovx+N groups received normal diet. Normal diet with additional 2% cholesterol was given to Ovx+ Cho (Adam et al. 2009).</div>\r\n<div>\r\n	Measurement of food intake and body weight</div>\r\n<div>\r\n	Food intake and body weight were measured at weekly intervals for four weeks in all the groups. Measurement of food intake was calculated by subtracting the total amount provided to the remaining amounts in the cage (Abdulla et al. 2011). Body weight was measured by using the electronic weighing scale. The increase in the body weight was calculated by deducting the final weight from the initial weight.</div>\r\n<div>\r\n	Blood pressure measurement</div>\r\n<div>\r\n	Prior to measure the blood pressure, the rats were made unconscious by inhalation of diethyl ether. Blood pressure of the unconscious rats were measured at the baseline and at the end of the experiment. It was measured by using the non-invasive tail-cuff method with sphygmomanometer technique using CODA system (a computerized monitor system) (Aida Azlina et al. 2009). Minimum of three measurements were taken consecutively and the average was then determined as a final measurment for SBP, DBP and mean blood pressure (Aida Azlina et al. 2009).</div>\r\n<div>\r\n	Statistical analysis&nbsp;</div>\r\n<div>\r\n	All data analyses were conducted using SPSS version 22. The normality of the data was determined by Kolmogorov&ndash;Smirnov test. The food intake and body weight changes among groups were compared using two-way ANOVA with Tukey post-hoc test. Statistical significance was defined as P &lt; 0.001. Data are expressed as means (&plusmn;SEM).</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Food intake and body weight</div>\r\n<div>\r\n	In the present study, the changes in the food intake and body weight of the animals following 4 weeks of consumption of 2% cholesterol diet was determined (Table 1). The food intake between Ovx+Cho and Ovx+N; Ovx+N and Sham groups were compared. It was shown that Ovx+Cho group (food intake 252.7 &plusmn; &nbsp; 9.17g; body weight 278.43 &plusmn; 13.9g) had a significant increase in food intake and body weight at the end of 4th week of the study compared to Ovx+N group (172.9 &plusmn; 9.67g); 247.43 &plusmn; 15.23g), respectively) (p&lt;0.001). Although, there was a significant increase in food intake in Ovx+N group (172.9 &plusmn; 9.67g) compared to sham group (152.9 &plusmn; 6.83g), the non-significant difference in body weights was found between Ovx+N (247.43 &plusmn; 15.23g) and Sham group (240.43 &plusmn; 12.23g) (p&gt;0.001). Less significant findings of food intake and body weights were observed between the groups at the different weeks of the study (week 0,1,2 and 3).&nbsp;</div>\r\n<div>\r\n	Systolic blood pressure measurement</div>\r\n<div>\r\n	The weekly systolic blood pressure was measured in normal and cholesterol &nbsp;fed groups with normal diet fed ovariectomised rats. The results were compared between the groups at the end of the study (i.e at 4th week of experimental period) (Figure 1). It was observed that Ovx+Cho group showed a significant increase in systolic blood pressure (128.2 &plusmn; 9.8mmHg) compared to Ovx+N group (115.1 &plusmn; 8.7 mmHg) &nbsp;(p&lt;0.001). No significant increase in systolic blood pressure was observed in the Ovx+N group compared to Sham group (112.2 &plusmn; 8.3 mmHg) &nbsp;(p&gt;0.001). There was no significant difference noted in the systolic blood pressure of Ovx+ Cho and Ovx+N groups; Ovx+N and sham groups at different week interval (week 0,1,2 and 3).&nbsp;</div>\r\n<div>\r\n	Diastolic blood pressure measurement</div>\r\n<div>\r\n	The weekly diastolic blood pressure was measured in normal and cholesterol diet fed ovariectomised rats and the results were compared between the groups at the end of the study (Figure 2). It was observed that the Ovx+Cho group showed a significant increase in diastolic blood pressure (95.2 &plusmn; 9.8 mmHg) compared to Ovx+N group (76.4 &plusmn; 9.2 mmHg) &nbsp;(p&lt;0.001). No significant increase in diastolic blood pressure was observed in the Ovx+N group compared to Sham group (77.5 &plusmn; 9.7 mmHg) (p&gt;0.001). The non-significant differences in diastolic blood pressure were observed at different week interval between the groups. &nbsp;</div>\r\n<div>\r\n	Mean blood pressure measurement&nbsp;</div>\r\n<div>\r\n	The weekly mean blood pressure was measured in normal and cholesterol diet fed ovariectomised rats and the results were compared between the groups at the end of the study (Figure 3). It was observed that the Ovx+Cho group had a significant increase in the mean blood pressure (135.2 &plusmn; 7.3 mmHg) compared to the Ovx+N group (96.7 &plusmn; 4.9 mmHg) &nbsp;(p&lt;0.001). No significant increase in mean blood pressure was observed in the Ovx+N group compared to Sham group(95.5 &plusmn; 5.3 mmHg) &nbsp;(p&gt;0.001). &nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Several clinical and experimental studies were carried out to investigate the complications that occurred in the postmenopausal state. Ovariectomised Sprague Dawley rats are used as the animals resemble the postmenopausal state of the human (Latour et al. 2001). With regard to the duration of the study, it is worth to know that there are several studies of cholesterol diet on short and long term period. According to the literature, short term period is presumed from 7 days to 4 weeks/less than 5 weeks and long term period is between 5-24 weeks of dietary cholesterol consumption. These durations are widely used in the atherosclerosis related cholesterol diet researches (Lichtenstein et al. 1994; Brown et al. 1991). &nbsp;</div>\r\n<div>\r\n	The present study used the ovariectomised rats to determine the changes in the blood pressure, body weight and food intake following 4 weeks of consumption of 2% cholesterol diet. The food intake and body weight was compared between Ovx+ Cho and Ovx+N groups: Ovx+N and sham groups. It was found out that there was a significant increase in the body weight and food intake in Ovx+Cho group compared to Ovx+N group (Table 1). The underlying mechanism related to the increased food intake in ovariectomised group fed with cholesterol is poorly understood. Yet, it was stated that cholesterol levels are strongly linked with the dietary intake. According to the literatures, the individual consuming more cholesterol in daily diet inclines to have increase food intake compared to the individuals with normal diet (Kresser 2013). Our findings agreed with the previous study which reported the significant increase in food intake in experimental rats within the first few weeks of ovariectomy (McElroy &amp; Wade 1987). The increase in the body weight following an increase in food intake might be most probably due to the gradual reduction in lean body mass and progressive fat accumulation in different body regions. Progressive fat accumulation induces oxidative stress and was proven to cause obesity, hypertension and cardiovascular diseases (Park et al. 2016). Previous study observed the weight gain in the ovariectomised rats are due to the oestrogen deficiency resulting an increase in the fat accumulation (Latour et al. 2001, Shinoda et al. 2002). However, the findings are not in line with the present results as Ovx+N group did not show any significance increase in the body weight compared to the sham group. It can be explained that oestrogen has less influence on the body weight in early onset of postmenopausal state. Based on the present study, the increase in body weight mainly reflect the consumption of cholesterol diet (Table 1). It is proved that the effect of oestrogen deficiency &nbsp;worsens with additional cholesterol intake.</div>\r\n<div>\r\n	Oestrogen deficiency causes a significant increase in the total cholesterol and triglycerides, which affects lipoprotein metabolism, platelet aggregation and vessel resistance (Gorodeski &amp; Gorodeski 2007; Jenkins et al. 2006). Previous studies observed that the intake of cholesterol rich food develops obesity and several cardiovascular complications in various animal models (Ali et al. 2006; Kennedy et al. 2010). This high cholesterol diet induces damage to the endothelium of large arteries and heart causing hypertension or increase in the blood pressure and subsequently, produce atherosclerosis resulting coronary heart disease (Ryou et al. 2012).</div>\r\n<div>\r\n	In the present study, Figure 1, 2 and 3 showed an increase in the systolic, diastolic and mean blood pressure of the Ovx+Cho group. It was observed that consumption of additional 2% cholesterol diet for 4 weeks caused significant changes in the blood pressure measurement. The oestrogen deficiency following ovariectomy and increase cholesterol intake are the predisposing factors of the obesity. In the state of obesity, the fat containing adipose tissue elevates circulating blood volume, plasma volume, and cardiac output, which dilates cardiac cavities and enhances in wall tension resulting in systemic and pulmonary hypertension and sudden death (Poirier et al. 2006). This incidence of hypertension frequently occurs in obese women with poor diet control, modern life style, existing family history and menopausal age (American Heart Association 2007). More than 73% of women aged between 65 to 74 years were reported to have high blood pressure (American Heart Association 2008; Yamakawa et al. 1995). Therefore, it is most likely that ovariectomised rats fed with 2% cholesterol diet (Ovx+Cho group) developed hypertension. No significant changes observed in Ovx+N group was most likely due to its early onset of postmenopausal state. Oestrogen deficiency is believed to effect the physiological changes in the prolonged duration. However, lack of oestrogen along with consumption of cholesterol diet may worsen the above findings even in the early onset. &nbsp;&nbsp;</div>\r\n<div>\r\n	In a routine research on &nbsp;cardiovascular disease, the ovariectomised rats fed with 2% cholesterol were used to develop the atherosclerosis animal model. The experiments related with this type of animal models are needed to be carried out for a prolong period (up to 24 weeks) (Lichtenstein et al. 1994; Brown et al. 1991) in order to achieve the significant outcomes of atherosclerosis or hypertension. Based on the present findings, it is proven that the use of 2% cholesterol diet induced a significant increase in food intake, body weight and blood pressure in ovariectomised rats over 4 weeks (short term period) following ovariectomy. This study showed that consumption of cholesterol diet in early onset of postmenopausal state leads to develop hypertension in the experimental animals. Moreover, this study highlighted the serious adverse effects of cholesterol on postmenopausal women. The development of hypertensive ovariectomised rat model in short term period with the consumption of 2% cholesterol diet is expected to give interesting results.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Based on the observations, we concluded that the hypertensive model is estabilshed with consumption of 2% cholesterol diet for short term period in ovariectomised rats. The model was easily developed, cost effective and less time-consuming. However, future studies are required to observe the detailed mechanisms related to the cholesterol diet and its potential hypertensive activity.&nbsp;</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(67631, 67617, '<div>\r\n	INTRODUCTION&nbsp;</div>\r\n<div>\r\n	Peripheral intravenous catheters (PIC) are widely used for therapy in current clinical practice. The Centers for Disease Control and Prevention (CDC) has stated that an estimated 250,000 catheter-related infections occur annually (O&rsquo;Grady et al. 2011).The peripheral intravenous catheters are mostly used for therapy to maintain electrolyte imbalance which include fluid infusion, blood transfusion, medications, and nutritional support (do Rego Furtado 2011). The insertion of PIC requires the breaking of skin barriers to allow insertion of temporary plastic tube into a vein. This will affect the hosts&rsquo; defenses against infection at the skin layers (Dougherty 2000). A previous study by Dychter et al. (2012) highlighted that nurses&rsquo; awareness of infusion-related complications can significantly affect patients&rsquo; outcomes and health care costs. The study also emphasized that complications of intravenous therapy are costly in terms of patient quality of life, morbidity, mortality, treatment expense, and extended length of hospital stay.</div>\r\n<div>\r\n	According to the World Health Organization (WHO 2009), routine hand washing in patient care is to remove dirt and organic material as well as micro-bacterial contamination acquired through contact with patients or the environment. A study by Hadaway (2012a) claimed that nurses&rsquo; lack of standardization in practice and knowledge of PIC care directly affects patients&rsquo; safety risks and outcomes. There were instances where patients experienced inadequate nursing care due to unsuccessful peripheral intravenous catheter insertion by inexperienced nurses. Another study by do Rego Furtado (2011) found that about 25% of unused peripheral intravenous catheters were placed in patients, or left there for a period of time. The standard guidelines and practice in the placement and maintenance of PIC is a major problem within the clinical setting. Several studies reported that PIC-associated bloodstream infections had endangered the hospitalized patients with prolonged hospital stay, increased cost, morbidity, and mortality (Woody &amp; Davis 2013). Although there are guidelines on insertion of PIC, the effectiveness of the implemented guidelines in nursing and allied health professions has received relatively little attention.&nbsp;</div>\r\n<div>\r\n	The risk factors of phlebitis due to peripheral intravenous catheter can be classified into four groups: catheter-related, drug-related, patient-related, and healthcare-related (Milutinović et al. 2015). It was reported that the incidence rate of infiltration was low but the rate of phlebitis was high. The study is consistent with findings by Maki and Ringer (1991) with phlebitis rate of 41.8%; Karadag and Gorgulu (2000) with 36.8%; and Uslusoy and Mete (2008) with 54.5%. According to Fitzgerald et al. (2012), although several physician-oriented researchers exist, the adherence and the effectiveness of implementation of guidelines in nursing context are relatively unknown.</div>\r\n<div>\r\n	Similar studies by Wilfong et al. (2011) and Hadaway (2012b) reported that nurses&rsquo; knowledge and early recognition of risk factors for the development of phlebitis could reduce complications. If the PIC is in situ without proper flushing and usage, it may lead to unnecessary complications and discomfort. Additionally, it would aggravate patients&rsquo; discomfort, stress and loss of confidence towards nurses. With that in mind, this study hoped to gauge nurses&rsquo; knowledge and practice in the prevention of PIC-associated bloodstream infections among hospitalized patients. Thus, main aim of the study was to determine nurses&rsquo; knowledge and practice towards the care of the PIC.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	Design</div>\r\n<div>\r\n	A descriptive cross-sectional design was conducted from April to June 2016. This hospital is a multi-disciplinary hospital with 365 beds and approximately 1,000 registered nurses. A convenience sampling of 90 respondents was recruited; 84 respondents (93%) had successfully completed the questionnaire and 6 (7%) respondents did not complete the questionnaire.</div>\r\n<div>\r\n	Data collection and instruments</div>\r\n<div>\r\n	The research instrument consisted of the respondents&rsquo; socio-demographic data which included age, years of experience, academic qualification, designation and department. A 26-item questionnaire was adapted and modified based on the Guidelines For The Prevention Of Intravascular Catheter-Related Infections from O&rsquo;Grady et al. (2011). It comprised 11 items related to knowledge of hand hygiene and aseptic technique, and another 15 items on practice of PIC care. Items were scored according to a 5-point Likert-type scale, which included strongly disagree = 1, disagree = 2, unsure = 3, agree = 4, strongly agree = 5. The questionnaire was piloted and Cronbach Alpha .80 was suggested that the items have relatively high internal consistency and reliability.</div>\r\n<div>\r\n	Ethical approval</div>\r\n<div>\r\n	The research was approved by the Research Ethics Committee of International Medical University (IMU) (BN 1/2016: PR-05) and the Director of the private hospital in Selangor. The privacy and confidentiality of each respondent was maintained and the respondents were given the rights to withdraw from participating in the study.</div>\r\n<div>\r\n	Data analysis techniques</div>\r\n<div>\r\n	Data analysis for this study was done using Statistical Package for Social Science (SPSS) version 22. The respondents&rsquo; demographic data were analyzed by descriptive statistics and independent t-test to identify the association between respondents&rsquo; demographic data with knowledge and practice towards the care of PIC.</div>\r\n<div>\r\n	RESULT</div>\r\n<div>\r\n	A total of 84 respondents completed the questionnaire. The respondents&rsquo; demographic data is shown in Table 1. There were 45 (54%) respondents aged between 21-26 yrs, and 39 (46%) who were more than 26 yrs. The majority of respondents (60; 71%) possessed Diploma in Nursing, and 24 (29%) held Bachelor of Nursing qualifications. A total of 63 respondents (75%) were from medical surgical wards while 21 respondents (25%) were from specialty units. A total of 32 respondents (38%) had working experience between 1-3 years; 21 respondents (25%) had 3-5 years working experience while 31(37%) of them had more than five yrs of working experience. There were 65 (77%) registered nurses and 19 (23%) charge nurses.</div>\r\n<div>\r\n	The findings in Table 2 showed higher mean score from specialty unit respondents, with knowledge (M = 49.19, SD = 3.44) and practice (M = 66.38, SD = 5.15), compared to the medical surgical wards with knowledge (M = 46.25, SD = 4.68) and practice (M = 63.17, SD = 4.63) towards the care of PIC, which was significant (p value = 0.010 and p value = 0.009, respectively). The study indicated a higher mean score of charge nurse respondents as compared with registered nurse with knowledge (M = 49.68, SD = 3.23); (M = 46.20, SD = 4.62) and practice (M = 67.11, SD = 4.84); (M = 63.06, SD = 4.61), which was significant (p value = 0.003 and p value = 0.001, respectively). There was no significant difference between respondents&rsquo; education qualification towards care of PIC with p &gt;0.05. However, the findings reported that respondents who possessed Bachelor of Nursing were deemed to score slightly higher in their knowledge and practice towards the care of PIC.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	Nurses&rsquo; roles and responsibilities are to ensure the delivery of optimum quality nursing care to hospitalized patients with PIC. The findings from this study indicated that nurses&rsquo; were knowledgeable and skillful in relation to the care of peripheral intravenous catheters in the hospital. Congruent with Woody and Davis (2013) study, it was found that insertion and maintenance of the PIC are considered essential nursing skills for all practicing nurses. O&rsquo; Grady et al. (2011) stated that it is important for nurses to have knowledge and good skills in nursing practice, in addition to frequent monitoring, checking and ability to identify complications associated with phlebitis, in order to prevent life-threatening consequences. According to do Rego Furtado (2011), nurses&rsquo; non-compliance to the guidelines on prevention of PIC-related phlebitis was associated with the catheter insertion technique and subsequent nursing care. The process of insertion, monitoring and assessing PIC should be an ongoing nursing activity. Therefore, the role of nurses is to constantly monitor the PIC site and patients&rsquo; feedback in order to ensure patients receive optimum care during hospitalization.&nbsp;</div>\r\n<div>\r\n	The findings from this study showed higher mean score of respondents from specialty units compared to the medical surgical wards in terms of knowledge and practice towards the care of PIC. A similar study by Deshmukh and Shinde (2014) reported that care of PIC demanded technical expertise, judgment skills and conscious, safe and efficient decision-making. It is a highly complex and specialized practice, and professionals who perform it should acquire theoretical and practical knowledge in continuing nursing education courses. This is supported by Glover (2000) who claimed in order to achieve continuous compliance and practice by nurses, better training, proper guidelines, and patient involvement are crucial. Furthermore, the infection control unit from hospitals should be stringent in monitoring and complying with the standard guidelines of PIC care. Registered nurses who are assigned as infection control link persons must ensure the infection control practices are implemented effectively in the clinical setting.</div>\r\n<div>\r\n	There were also significant differences between charge nurse and registered nurses&rsquo; knowledge and practice in this study. A charge nurse is considered an experienced nurse who has fulfilled the criteria of working more than five yrs with an excellent Key Performance Index (KPI) as required by the hospital. In accordance to the findings, charge nurses were deemed to possess better knowledge and higher practice score due to their expertise and ability to make appropriate decisions in difficult situations. This is congruent with Benner&rsquo;s (1984) 5 Stages of Clinical Competence. At Stage 1, a novice nurse is newly graduated at the initial stage with very limited ability to predict what might happen in a particular patient&rsquo;s situation. In Stage 2, as an advanced beginner, a nurse gradually acquires experience but still lacks knowledge. At this point, the nurse is guided by rules, oriented to complete nursing tasks, and requires mentor assistance in assessing difficult situations. At Stage 3, a competent nurse does not have the speed and flexibility of proficient nurses, but possess a certain level of mastery and can rely on advanced planning and organizational skills after two to three years of working experience. In Stage 4, a proficient nurse is capable to see situations as &ldquo;wholes&rdquo; rather than parts. It begins from three to five working years and the nurse possess the ability to learn from experience is able to modify plans in response to different events. Finally, at Stage 5, an expert nurse no longer relies on analytical principles such as rules and guidelines to connect understanding of the situation to an appropriate action. The nurse would require more than five yrs of working experience to focus on the most relevant problems. Therefore, the findings of this study revealed that charge nurses were deemed to possess higher scores of their knowledge and practice, as supported by Benner&rsquo;s (1984) theory.&nbsp;</div>\r\n<div>\r\n	Respondents&rsquo; academic qualification also has implications on their knowledge and practice of PIC care. Those with Bachelor of Nursing reported a higher score in knowledge and practice compared to Diploma of Nursing graduate students although statistically it was insignificant. The difference in prerequisite entry qualifications for Bachelor of Nursing and Diploma in Nursing programmes may be the contributing factors. The Bachelor of Nursing programme required students to have A-level, Malaysian Higher School Certificate (STPM) or Matriculation qualifications, whereas the Diploma of Nursing programme caters to Malaysian Certificate of Education (SPM) graduates. According to Lee et al. (2009), nurses who have higher education qualifications are better prepared to update themselves with the latest nursing practices, including the prevention of PIC complications. In the Malaysian nursing profession, there are two categories of registered nurses, namely Diploma in Nursing graduates and Bachelor of Nursing graduates. The results of this study suggest that nurses with Bachelor of Nursing qualifications are better off in terms of knowledge and skills but this difference was not statistically significant. Perhaps a larger and more robust study would be able to confirm this.&nbsp;</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, majority of the nurses possessed adequate knowledge and practice towards the care of PIC. The specialty unit and charge nurse respondents were deemed to possess better knowledge and practice towards the care of PIC in the hospital. The respondents&rsquo; age and years of experience revealed no significant difference in their knowledge and practice towards care of PIC in this private hospital. Nurses should be encouraged and given the opportunity to attend continuing nursing education in order to gain more exposure to the latest nursing practice updates and increase their knowledge about PIC care.</div>\r\n<div>\r\n	Acknowledgment</div>\r\n<div>\r\n	The authors would like to express greatest appreciation to Dr Jeetha Muthumanikam and Ms Chong Nyok Ngoh for their contribution to the research.</div>\r\n', 1),
(67654, 67640, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Normal breasts or mammary glands produce milk. In normal cell cycles, proliferation and regression are controlled by hormones and growth factors. Abnormal cellular changes progress from proliferative disease to atypical hyperplasia to carcinoma in situ to invasive breast cancer. While the majority of breast cancers are adenocarcinomas occurring in the upper outer quadrant, breast cancer is a complex and heterogeneous group of diseases with distinct morphologic and molecular features (Foxson et al. 2011; Cado &nbsp;et al. 2013; Watanabe et al. 2015). Over the past century there has been a dramatic increase in the pathological abnormality of the cases which is referred to as cancer. &nbsp;An American Cancer Society (2015) reported that cancer is best defined as diseases characterized by uncontrollable growth of abnormal cells which could spread to other parts of the body, and potentially be lethal.</div>\r\n<div>\r\n	In Malaysia, the incidence with regard to age suggested that the breast cancer increased steadily from the age group of 30 yrs and the peak was from 40 to 59 yrs. In contrast, in Western countries a high proportion of patients presented with an advanced-stage of cancer. A Malaysian women&rsquo;s cumulative risk of breast cancer in her lifetime was in the ratio of 1:20 with the highest risk in Chinese (1:14) (National Cancer Registry Malaysia 2006; Hisham &amp; Yip 2004). Breast cancer could be caused by a diversified modifiable and non-modifiable risk factors which include gender, age, history of benign breast or related diseases and reproductive factors such as menstrual history, first pregnancy&rsquo;s age, lifestyle, hormonal, and genetic factors.&nbsp;</div>\r\n<div>\r\n	Genetic factors in the pathogenesis of breast cancer can be ascribed to be inherited susceptibility. Three genes (p53, BRCA1, and BRCA2) were identified, which may alter the formation of tumour suppressor genes. In breast cancer p53 mutation/alteration are reported in &nbsp;50% of primary carcinomas (Callahan 1992; Muller &amp; Vousden 2013; Bertheau et al. 2013). The p53 is a tumour suppressor gene which responds to the DNA damage that induces the pathways for cell apoptosis, cell cycle arrest, and DNA repair. The function is to maintain the genetic integrity of the cells resulting p53, as one of the most studied proteins in oncology (Steele &amp; Lane 2005). Previous study by Thompson, (2014) reported that the abnormalities observed in the p53 gene are common and could be the key events in the pathogenesis of breast cancer.</div>\r\n<div>\r\n	The Clinical Practice Guideline (CPG), Ministry of Health Malaysia (2003), emphasizes the management of breast cancer through reinforcing those women whom genetically related to higher propensity of deleterious mutations in BRCA1, BRCA2, and TP53 genes. They are advised for genetic counselling by geneticist; and genetic testing prior to their early detection. &nbsp;Currently, there is limited scientific evidence which proposes the evaluation of other genes or specific loci in common clinical setting of the Ministry of Health for genetic testing (MOH &amp; AMM 2003). In diagnosing p53 expressions and relationship with breast cancer occurrence, an IHC analyses must be a warranted design by utilizing a statistical analysis. However, an IHC analysis cannot be ruled out as a useful indicator of prognosis in clinical applications of &nbsp;breast cancer (Bartley &amp; Ross 2002; Rolland et al. 2007). &nbsp;Detection of p53 protein by IHC has been widely used as a surrogate marker for p53 mutations. Even though, the lacking of p53-binding domain was noted, the physiological activities were dependent on p53 in which, the binding to wild-type MDM2 was responsible for cancer pathophysiology (Inoue &nbsp;et al. 2016). Secondary cancer prevention is a set of intervention that uses screening tests or examinations which leads to the discovery, control of cancerous and precancerous processes. A study by &nbsp;Loud et al. (2002) stated that pathophysiological alteration and possible counteraction mechanism of breast cancer should be first well understood.</div>\r\n<div>\r\n	In the present study, the main aim was to determine the specific patterns in breast cancer (BC) risk factors by p53 status and identifying the way forward, in screening for early detection. A study by Maas et al. (2016) stated that 30 yrs women in the United States may develop invasive breast cancer at the rate of 11.3% by the age of 80 yrs. In addition, the proposed model which accounted for the risk factors depicted 4.4% - 23.5% of the average absolute risk in bottom and top deciles of the risk distribution in women. Apparently, women who fell in the lowest and highest deciles of non-modifiable risk factors accounted for 5th and 9th percentile range of the risk distribution related to modifiable factors and were recorded as 2.9% - 5.0% and 15.5% - 25.0%, respectively. Consequently, women who were at the highest decile of risk pertaining to non-modifiable risk factors, low BMI, non-alcoholic and non-smoker, and never used MHT had higher risks comparable to general population of average woman. According to Maas et al. (2016) that highlighted traditionally, the etiology and pathophysiology of breast cancer were similarly reported globally. However, the differences in genetic materials of Asian women could possibly triggers the difference interaction mechanism with environmental exposure. &nbsp;This may include reproductive risk factors and dietary factors that may be involved in alteration of the susceptibility towards beneficial or deleterious effects of these exposures. Hence, it is suggested that the relevant contributing risk factors may contradict between Western and Asian populations. Biomarker data would enable in strengthening the link between risk factors and breast cancer as shown in epidemiological studies. Subdividing breast cancer cases by p53 protein expression and searching for important patterns in risk factors of breast cancer by p53 status could help to narrow the search for specific p53 mutations. Therefore, this provides the impetus for the present study to investigate the prevalence of breast cancer within epidemiological risk factor and association with p53 protein expressions in breast cancer tissue specimens in premenopausal women.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	DESIGN</div>\r\n<div>\r\n	Demographic and risk factor profiles data were taken from breast cancer tissue registries and paraffin embedded breast tissue samples were then detected for p53 protein expression using immunohistochemistry technique. The results of p53 protein were then compared with risk factor profiles available from patient&rsquo;s registries.&nbsp;</div>\r\n<div>\r\n	Breast cancer tissue sampling</div>\r\n<div>\r\n	A total of 111 breast carcinoma tissue samples in paraffin blocks were obtained from female patients who were diagnosed between June 2005 and April 2009. The age of the patients ranged from 27 to 82 yrs (mean age of 54 yrs). These paraffin embedded breast tissue samples were randomly picked from a listed serial number of paraffin blocks available from the computed registry of the Pathology Laboratory, Hospital Tuanku Jaafar, Seremban, Negeri Sembilan. A retrospective review of the study population was available from the patients clinical histopathology report (Form Perub. J.P. 06) which included patient&rsquo;s age and tumour grade status and several other risk factor profiles.</div>\r\n<div>\r\n	Data collection and instruments</div>\r\n<div>\r\n	Demographic data and risk factors profile were taken from the patients&rsquo; histopathology laboratory reports, mammography and surgical outpatient follow-up case reports. &nbsp;Based on patients address they were residents of Negeri Sembilan and being referred to Hospital Tuanku Jaafar, Seremban. Patient&rsquo;s age was defined as the age of cancer diagnosis.</div>\r\n<div>\r\n	ETHICAL APPROVAL</div>\r\n<div>\r\n	Ethical approval was obtained from Research Ethics Committee of both Universiti Teknologi MARA (UiTM) and also MOH Research &amp; Ethics Committee (MREC) of the Ministry of Health (MOH) (NMRR 8-16-1771).&nbsp;</div>\r\n<div>\r\n	DATA ANALYSIS TECHNIQUE</div>\r\n<div>\r\n	Immunohistochemical (IHC) Staininig</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Paraffin embedded tissue sections, 5 &mu;m thick, were mounted on silanized slides and fixed for 12 mins, The paraffin embedded slides were processed for IHC staining and evaluated for expression of p53. The histological type of breast cancer selected randomly included invasive and non-invasive carcinoma and other benign breast diseases. Grading of breast tumour was determined using modified Bloom and Richardson (1957) criteria &nbsp;as stated in the histopathology report.&nbsp;</div>\r\n<div>\r\n	IHC was performed using the DakoCytomation kit (EnVision + System-HRP; DakoCytomation Denmark A/S, Glostrup, Denmark). Paraffin-embedded tissue sections from all samples were stained with p53, accordingly. Tissue sections were de-paraffinized and hydrated prior to antigen retrieval by microwave, followed by inhibiting the endogenous peroxidase activity by 5 mins incubation and rinsed with distilled water. Following these step, slides were treated for 10 mins with primary antibodies clone DO-7 (N158187, Dako) and optimum dilutions were determined according to experiments. Slides were then rinsed with Tris buffered saline solution (TBS) with 0.05% plus Tween 20 (S196630, Dako) twice for 3 mins each time. Sections were incubated with REAL&trade; Envision Polymer (K4065, Dako) as peroxidase labelled polymer to the primary antibody for 10 mins each time. Antigen was visualized with DAB (3, 3&rsquo; diaminobexidine) chromogen (K4065, Dako) plus diaminobexidine substrate buffer (K4065, Dako) preparation and counter-stained with haematoxylin staining. The incubation protocols were conducted at room temperature and the tissue sections were carefully washed within the incubations series. In every experiment, in-house positive p53 tissue sections with one positive control and one negative control were included.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Semiquantitation of p53</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	For overexpression of p53 protein in samples, the staining intensity on tissues is scaled between 0 to +3 scores. Relatively, tissue section which did not exhibit brownish nuclear reactivity in tumour cells was recorded as 0. Tissue section which occasionally showed positive nuclear staining was scored as +1, provided the total percentage of positivity in tissue section was &lt; 20%. Any tissue section which tumour cells exhibited positive nuclear staining in between 20% - 80% was given score of +2. For the +3 score, the scale was given to tissue section which depicted positive nuclear staining in &gt; 80% of tumour cells, and the scales of +1, +2, and +3 were recorded as p53+. Microscopic examination observed using 10X and 40X magnification lens field. IHC was confirmed positive if the reaction was visualized by Dako REALTM DAB+ (3, 3&rsquo;-diaminobenzidine tetrahydrochloride in organic solvent) substrate-chromogen solution which results in a brown-colored precipitate at the antigen site of p53 protein (Figure 1).</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	The diagnostic age groups among breast cancers was elaborated in Table 1. The age at diagnosis ranged between 27 to 85 yrs. The peak age of diagnosis was between 45 and 55 yrs, &nbsp;with the mean age of 54 yrs (S.D 12. 91) while the mode age was 46 yrs in the premenopausal age group. In Table 1, revealed that the majority of p53 (+) protein expression was found in the age groups above 41 yrs. The results indicated that majority (n=41/111) who were more than 41 yrs, had a positive p53 (+) protein category. In addition, minority (n=3/111) of the respondents were aged 41 yrs and less and also had positive p53 (+) protein category (n= 3/111). However, the Fishers exact test, indicated that, there was no statistically significant correlation between age category and p53 protein category (&chi;2 (1) = 0.78, p =0.52), and there was no correlation strength (Cramer&rsquo;s V coefficient = 0.08) and also not significant (p =0.37) (Table 1).</div>\r\n<div>\r\n	To strengthen the correlation findings of the p53 protein expression and the unmodifiable age as risk factor ,the estimation study was conducted. Table 2 showed the result of risk estimate of p53 protein and age group. The results reported that the risk factor was reduced by 0.66 (or 66.5%) for women aged &le; 41 yrs in expressing p53 (+) protein, Risk estimate = 0.66, 95% confidence interval (CI): 0.24- 1.79), compared to the women aged &gt; 41 yrs. Therefore, &nbsp;age group did not have any significants (p&gt;0.05) in overexpressing &nbsp;p53 (+) protein. The risk estimate showed probability of p53 (+) protein being overexpressed in the age group less than 41 yrs was 0.66 times less likely compared to the age group above 41 yrs.&nbsp;</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The risks pattern of breast cancer among women could be identified by the overexpression of p53 in breast cancer tissue samples which showed the presence of mutant p53. The findings indicated that this protein would not be degraded or metabolized in the tissues and continued to exert its original function. The present study reported inhibition of the apoptosis induction that further triggered the uncontrollable growth of tissues that resulted in tumour formation. Hence, becoming aggressively malignant and invasive. The highest percentage of expressing p53 (+) in breast cancer tissue was found in the age group &gt; 41 yrs. However, the percentage of p53 (+) protein overexpression increased with the age group of this study. The majority of the respondents (36.9%) of p53 (+) overexpression were observed in the breast cancer tissue of respondents within the age group of &gt; 41 yrs, while only (2.7%) of p53 (+) overexpression was observed within the age group &lt; 41 yrs. &nbsp;</div>\r\n<div>\r\n	Our study reported that a total of 111 participants diagnosed with breast cancer between June 2005 and April 2009, with age groups &nbsp;within the range of 27 to 85 yrs and &nbsp;mean age of 54 yrs upon diagnosed as breast cancer. &nbsp;Their median age was 52 yrs and mode age of 46 yrs. Congruently supported &nbsp;with several &nbsp;studies (Lim et al. 2008; Musa et al. 2011). Another study Lim et al. (2008) had similar results to the present &nbsp;study in which &nbsp;the median age at upon diagnosis was 52-53 yrs and &nbsp;age standardized rate (ASR) was between the age group 50-59 yrs.&nbsp;</div>\r\n<div>\r\n	The participant&rsquo;s mode age with breast cancer was within 46 yrs. According to Hisham &amp; Yip (2004) and Toh &nbsp;et al. (2008) &nbsp;similar findings were found that &nbsp;nearly 50 % of breast cancer cases occurred &nbsp;in women under the age of 50 yrs. Furthermore, Hisham and Yip (2004) showed that predominant age group &nbsp;between 40-49 yrs and a high proportion were in the advanced stage of the disease. In addition, Leong et al. &nbsp;(2010) reported a median age of 52 yrs for breast cancer occurrence showed a salient difference in peak age between 40-50 yrs in Asian countries and between 60-70 yrs in Western countries &nbsp;However, a study by Rohan et al. (2006) stated that participant&rsquo;s age &nbsp;may be a prognostic factor in determining the probability of increasing metastatic potential in breast cancer and it is supported with overexpression of p53 (+) protein that could be &nbsp;reflected to a more advanced stage of progression of the breast cancer.</div>\r\n<div>\r\n	Significantly, this study may suggest that p53 (+) overexpression could be an indicator for the likelihood of occult alterations existence in breast cancer tissues with non-expressing p53 (-) protein in a younger age group, whereby this predictor may provide a valuable indication for earlier detection of breast cancer occurrence. Earlier studies by Bertheau et al. (2008) and Agrawal et al. (2009) showed results ranging from 14-58% of p53 (+) protein overexpression in breast cancer tissues. The findings from this present study reported that overexpression of p53 (+) protein was found in 40% (44/111) of the overall breast cancer cases. Notwithstanding, there were strong similarities between the current study and earlier scientific works in terms of the overexpression of p53 signified in approximately 14-58 % of diagnosed breast cancer cases.</div>\r\n<div>\r\n	Adjustment of age in the present study would enable to reduce the risk factors of age group &lt; 41 yrs to express p53 (+) protein. The risk estimation showed probability of p53 (+) protein being overexpressed in the age group less than 41 yrs was 0.66 times less likely compared to age group above 41 yrs. The participants&rsquo; age groups were related to a non-statistically significant increase in the risk of p53 protein. The increased odds (OR 1.85) of p53 (+) protein expression with age groups did not reach statistical significant as indicated, with (p &gt; 0.05). The present study concurred with Erdem et al. (2005) who failed to demonstrate any association between p53 protein expression and age. However, this present study, indicated the correlation p53 protein and age groups did not reach a statistical significance (p &gt; 0.05), the frequency rate, of p53 (+) protein expression among women &gt; 41 yrs compared to &lt; 41 yrs, was 36.9% (41/111) and 2.7% (3/111), respectively.&nbsp;</div>\r\n<div>\r\n	The finding from the present study showed that participant&rsquo;s age groups can be a predictor of risk factors for breast cancer among older women. Congruently supported by previous studies Worsham et al. (2007), Etebary et al. (2002), and Dupont and Page (1989). The alteration and mutation in P53 gene, or overexpression of p53 protein as a secondary marker in &nbsp;the mutation pathways would likely be considered &nbsp; as a potential prognostic factor or biomarker for numerous human malignant tumours, particularly breast cancer in which has been recognized with worse prognosis (Bergh et al. 1995; Kikuchi et al. 2013).&nbsp;</div>\r\n<div>\r\n	Limitation of this study could be attributed with the suboptimal methods in determination of &nbsp;p53 status due to the small sample size which &nbsp;did not allow more reliable assessment to associate with participant&rsquo;s age factor and p53 protein. &nbsp;However, the analysis of p53 protein expression was carried out in the absence of knowledge regarding case-control status which unlikely &nbsp; contributed to the differential bias in the current evaluation of p53.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion with &nbsp;or without overexpression of p53 protein women above 41 yrs were at a greater risk diagnosed with breast cancer. The investigation of p53 (+) overexpression in relation to participant&rsquo;s age group in the present study revealed that there was fluctuation in the frequency of the detection rate of p53 protein. This limitation could be contributed by the low statistical power of the clinical data with reference to the potential predictive value of the p53 protein which are still conflicting.&nbsp;</div>\r\n<div>\r\n	ACKNOWLEDGEMENT&nbsp;</div>\r\n<div>\r\n	This study was funded by e-Science Grant from Ministry of Science, Technology and Innovation Malaysia (MOSTI). The authors wish to extend their appreciation to &nbsp;Institute of Research Management &amp; Innovation (IRMI), UiTM for permission to publish this work, Assoc. Prof. Dr. Zuridah Hassan, Ministry of Health for ethical approval and permission to collect samples from Hospital Tuanku Jaafar, Seremban, pathologists and staff of Histopathology Department, Hospital Tuanku Jaafar, Seremban for their cooperation.&nbsp;</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(67662, 67648, '<div>\r\n	Introduction&nbsp;</div>\r\n<div>\r\n	Microvascular complications of diabetes dominated as the leading cause of preventable blindness in working aged people in many developed countries (Cheung et al. 2010). Over the past 30 years, pan retinal photocoagulation (PRP) has remained as the gold standard for treating ischemic and neovascular diseases of the retina. &nbsp;PRP reduces the risk of severe visual loss by 50% in patients with high risk proliferative diabetic retinopathy (DRSRG 1978).</div>\r\n<div>\r\n	The principle by which the laser exerts its therapeutic effects relies on the destruction of the outer retina. This reduces the oxygen demand while maintaining a constant oxygen input. Laser burns may also serve as physiological windows through which oxygen diffuses from the choroid, increasing vitreous oxygenation and induced vasoconstriction (Velez-Montota et al. 2010). However, despite the efficacy for preventing severe visual loss, laser PRP is often associated with substantial ocular side effects, such as difficulty with light&ndash;dark adaptation, a slight decrease in vision, and peripheral visual loss (Pender et al. 1981; Henson &amp; North 1979).</div>\r\n<div>\r\n	The energy of a conventional argon laser is transformed to heat at the level of the retinal pigment epithelium (RPE) and the outer retinal layer. However, longer laser wavelengths will penetrate deeper than the RPE and cause more destructive changes to the retina, RPE, and choriocapillaris (Maeshima et al. 2004; Nagpal et al. 2010). Pattern scanning laser (PASCAL) is a novel device that has radically changed traditional laser parameters. Normal conventional laser requires an average duration of average of 100 ms compared to only 20 ms for PASCAL. Velez-Montoya et al. (2010) demonstrated short duration of PASCAL laser allows lower total energy used to produce a desired burn. This consequently limits the spreading of choroidal heating and elicits a more uniform energy distribution with less inner retinal injury (Muqit et al. 2010a). Another major advantage of PASCAL is the ability to distribute the laser treatment over a wider area of the retina in a single session with less collateral damage (Nagpal et al. 2010). At the same time, retinopathy regression was similar to the conventional argon green laser (Nagpal et al. 2010).</div>\r\n<div>\r\n	Argon laser has long since been known to cause a significant reduction in RNFL thickness over time (Kim &amp; Cho 2009). Transient thickening post-PRP with the conventional argon laser has also been described by many authors (Kim &amp; Cho 2009). However, the effect of PASCAL on peripapillary RNFL thickness has not been well described. Therefore, this study was designed to compare the immediate changes in peripapillary RNFL thickness post-PRP between conventional argon lasers and PASCAL.&nbsp;</div>\r\n<div>\r\n	materials and Methods</div>\r\n<div>\r\n	This was a hospital based, randomized clinical trial study. It was conducted at UKMMC, Kuala Lumpur, Malaysia. Ethical approval was obtained from the Research and Ethics Committee of Faculty of Medicine, Universiti Kebangsaan Malaysia. The study was conducted from March 2011 to December 2012. Written consent was obtained from all the participants.&nbsp;</div>\r\n<div>\r\n	Participants</div>\r\n<div>\r\n	Recruited patients included those with proliferative diabetic retinopathy (PDR) or very severe non-proliferative diabetic retinopathy (NPDR) requiring PRP laser treatment. An experienced ophthalmologist clinically graded the stages of diabetic retinopathy and diagnosis was made based on the early treatment diabetic retinopathy study (ETDRS) classification. Patients were randomized into two groups based on the treatment received: conventional argon laser or PASCAL.&nbsp;</div>\r\n<div>\r\n	Inclusion criteria were: i) type 2 diabetes mellitus with very severe NPDR or PDR requiring laser treatment; ii) clear media without clinically significant amount of cataract; iii) age of diabetes mellitus diagnosis &gt;35 years; iv) and best corrected vision of 6/18 or better to ensure good signal strength on optical coherence tomography (OCT). Exclusion criteria were: i) type 1 diabetes mellitus; significant media opacity; ii) co-existing glaucoma; iii) &nbsp;previous laser treatment or eye surgery; iv) degenerative myopia or uveitis; v) or any history of other vascular diseases, such as retinal artery or vein occlusion.&nbsp;</div>\r\n<div>\r\n	Data collection</div>\r\n<div>\r\n	Complete eye examinations, fundus photographs,and OCT (Carl Zeiss Meditec Inc., Dublin, CA) measurement of the peripapillary RNFL were performed prior to laser and post-treatment at two and four months. A 3.4 mm peripapillary scan mode was used to measure peripapillary RNFL thickness. Patients with good signal strength on OCT (&gt;5) were included.&nbsp;</div>\r\n<div>\r\n	A single, medically trained person performed the PRP. A total of 900 to 1,500 laser spots were generated at each sitting. Time interval of one to two weeks was maintained between two sittings. A laser spot size of 200 or 300 &micro;m was used for both modalities. Pulse duration of 20 ms was used for PASCAL and 100 ms for the argon laser. The power was adjusted to achieve gray burns. Spots were placed one spot distance apart. Topical anesthesia was used in all eyes. Among completing the treatment, total number of laser spots in each eye ranged from 2,000 to 3,000 shots. Treatment was considered complete once there was clinical evidence of regression of neovessels, and intraretinal hemorrhage, improvement in venous dilatation, and no new pre-retinal bleed. Pallor of the disc was unlikely to be observed at four months post-laser. Therefore, it was not used as an indication of disease regression.&nbsp;</div>\r\n<div>\r\n	Ethical approval</div>\r\n<div>\r\n	The research was approved by the Ethics committee of UKMMC (FF-2010-347). The privacy and confidentiality of each individual was maintained and the respondents were given the rights to withdraw from participation.</div>\r\n<div>\r\n	Data analysis</div>\r\n<div>\r\n	The statistical analysis in this study was presented descriptively. Univariate, bivariate, and multivariate analyses were performed using the Statistical Package for Social Sciences (SPSS) version 20.0 (IBM Corp, Armonk, NY, USA). The differences in RNFL thickness were analyzed using paired t-tests (normal data), whereas the Chi-square test was used for categorical data and general linear models (repeated measurement analysis of variance [ANOVA]) were used for multivariate analysis. A p-value less than 0.05 was accepted as statistically significant.</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Demographic information</div>\r\n<div>\r\n	A total of 32 subjects were recruited for the study; 16 patients in the argon group and 16 patients in the PASCAL group, and 27 patients had PDR and five had very severe non-PDR. The mean age for the argon group was 56.44 &plusmn; 7.87 yrs and Pascal group was 54.00 &plusmn; 7.94 yrs (p=0.390). The overall age range of both groups was 41 to 78 yrs. The duration of diabetes was comparable between the two groups with a mean duration for patients receiving argon laser of 7.31 &plusmn; 4.81 yrs and for the PASCAL group 6.75 &plusmn; 3.75 yrs (p = 0.71). Table 1 summarised the demographic data and clinical characteristics of the study population.&nbsp;</div>\r\n<div>\r\n	Peripapillary retinal nerve fiber layer thickness changes in the conventional argon group&nbsp;</div>\r\n<div>\r\n	Average RNFL before laser treatment was 108.64 &plusmn; 17.70 &mu;m, and a slight increase was noted at two months post-treatment (112.64 &plusmn; 17.05 &mu;m; p=0.127). Baseline RNFL before treatment was used as comparison. Subsequently, at four months, average RNFL was 106.55 &plusmn; 15.77&micro;&mu;m (p=0.457). Analysis of the individual quadrants (temporal,nasal,superior and inferior) showed similar transient thickening of the RNFL at two months, except for the nasal quadrant. Superior RNFL measurements prior to laser, at two and four months were 127.75 &plusmn; 22.12 &mu;m, 132.38 &plusmn; 21.99 &mu;m (p=0.200), and 126.69 &plusmn; 20.36 &mu;m, respectively (p=0.795). Nasal quadrant thickness was the only quadrant that showed a slight reduction in thickness from baseline after laser treatment: 86.31 &plusmn; 21.33 &mu;m, 79.58 &plusmn; &nbsp;24.41 &mu;m (p= 0.404), and 80.63 &plusmn; 19.49 &mu;m, respectively (p=0.366).&nbsp;</div>\r\n<div>\r\n	Although the transient thickening at two months was not statistically significant, there was a consistent trend of thickening observed in all quadrants except for the nasal region (Table 2, Figure 1). These changes were temporary and RNFL in all quadrants recovered to baseline thickness at four months.&nbsp;</div>\r\n<div>\r\n	Peripapillary retinal nerve fiber layer thickness changes in the PASCAL group&nbsp;</div>\r\n<div>\r\n	Conversely in the PASCAL group, RNFL showed continuous significant thickening at two and four months post laser treatment. Significant changes were found for the average RNFL at two and four months, inferior RNFL at four months and nasal quadrant at two and four months. Average RNFL thickness before laser treatment was 99.26 &plusmn; 13.96 &mu;m, 105.52 &plusmn; 17.39 &mu;m at two months (p=0.003), and 107.20 &plusmn; 12.14 &mu;m (p = 0.001) at four months (Table 3, Figure 2).</div>\r\n<div>\r\n	Overall, in the PASCAL group, increasing trend of RNFL thickness occurred in all quadrants with time. Compared to the argon group, RNFL thickness &nbsp;failed to return to its baseline level at four months.</div>\r\n<div>\r\n	Discussion&nbsp;</div>\r\n<div>\r\n	Histologically, Blumenkrantz et al. (2006) reported application of 100 ms threshold level of 514 nm argon green laser in rabbit eyes produced a complete loss of the photoreceptors as well as hyperplasia of the pigment epithelium). High powered lasers cause transmission of heat to adjacent tissue causing destructive effects and inflammatory response immediately after laser treatment. This transmitted effect predisposes patients not only to macular edema due to transient choroidal thickening, but, in the long run, increases the risk of developing epiretinal membranes (Kim &amp; Cho 2009). Moreover, the end point laser marks tend to exaggerate with time, which correlates well with reported histological data (Maeshima et al. 2004). Such effects are well tolerated in older age groups, but with the increasing number of young diabetics (Geiss et al. 2014), these side effects may significantly affect their quality of life.&nbsp;</div>\r\n<div>\r\n	The aim of new laser technologies is to achieve retinal photocoagulation that causes healing responses by selectively targeting the RPE with minimal photoreceptor loss and scar expansion. PASCAL uses a special scanning pattern to permit rapid photocoagulation, and thus reduces burn session time, improving patient comfort as well as ensuring excellent physician performance and efficiency (Blumenkranz et al. 2006; Muqit et al. 2012). PASCAL technology is a semi-automated pattern generation method using short laser pulse durations of typically 20 ms (five times shorter than conventional systems). Moreover, argon lasers and PASCAL have been proven to be equally effective for treating proliferative diabetic retinopathy (Naqpal et al. 2010). OCT observations by Inagaki et al. (2012) showed that the PASCAL laser reduces the vertical and horizontal spread of laser energy, thus producing shorter defect in the inner/outer segment line compared to the conventional laser group.</div>\r\n<div>\r\n	Apart from limited injury to the entire retina, PASCAL laser scars were not found to enlarge with time (Muqit et al. 2010b). Histopathology evaluation by Paulus et al. (2013) reported preservation of the inner nuclear layer of RNFL post PASCAL laser. Previous OCT study, describe transient RNFL thickening occurs immediately after argon laser treatment and may last for up to 6 months (Lee et al. 2013). As opposed to our study, transient thickening in our study did not last beyond four months. The cause of this transient thickening remained uncertain although many authors postulated the presence of upragulated inflammatory mediators occur post laser treatment. &nbsp;Nonaka et al. (2002) reported increase number of leukocytes and vascular permeability, which resulted in retinal edema post-lase).</div>\r\n<div>\r\n	To the best of our knowledge, there is no previous study reporting changes in peripapillary RNFL thickness after PASCAL laser in diabetic patients, let alone comparing conventional argon to PASCAL laser. It is interesting to note that, in the present study, the PASCAL laser produced more thickening of the peripapillary RNFL compared to the argon group immediately after multi-session PRP. Also, the increased thickening continued to progress even at four months post treatment. The common argon laser setting for retinal ablation was 100&ndash;200 ms duration and the power used ranged from 150&ndash;350 mW. The PASCAL laser, on the other hand, uses higher power to deliver the same amount of energy with extremely shorter pulse duration of 10 ms (Lee et al. 2013). Therefore, in general the amount of energy received by the lasered area is lower compared to the conventional argon treatment (Velez-Montoya et al. 2010). The possible explanation at this point is the probability of higher power used during PASCAL laser induces greater inflammatory responses compared to the argon laser. Paulus et al. (2011) in another study, described the histological changes of retina in rabbits post pascal laser. Spot size used was less than 60um with lacking of description on the power used. The authors observed accumulation of oedema between the outer segment and RPE layer. Resolution was seen as early as one month with persistent preservation of the inner retinal layer (Paulus et al. 2011). Similar observation might occur in vivo. However, direct comparison with our findings might not be feasible. Despite uncertain explanation for the persistent peripapillary retinal swelling in our study, it is important to note that the retinal cells react differently with PACAL laser. &nbsp;Evaluation of peripapillary RNFL in such cases immediate post laser will not represent the actual RNFL thickness and has to be evaluated with caution.&nbsp;</div>\r\n<div>\r\n	Although the limitations of this study were due to the short duration of follow-up, our findings are significant enough to indicate a difference in tissue response between these two treatment modalities. Longer follow-up in larger study scale might help to determine the onset of RFNL thickening resolution that is known to occur with time. Another limitation involved the use of time domain OCT which is the earlier version of biomicroscope. Time domain OCT was the only machine available during the study period. Therefore, future study design the latest spectral domain OCT will be able provide additional biomicroscope changes and offer better explanation for such changes.&nbsp;</div>\r\n<div>\r\n	In conclusion, transient thickening of the RNFL occurs post-PRP in both conventional and PASCAL laser patients. However, the PASCAL laser seems to produce more cellular reaction, hence increases the RFNL thickness to a greater extent than the argon laser group. This is important as RFNL measurements have been incorporated as one of the gold standards of glaucoma evaluation. Important events, such as laser treatment to the eye and type of laser used worth of consideration during OCT evaluation of RNFL thickness in such cases.</div>\r\n<div>\r\n	Acknowledgements</div>\r\n<div>\r\n	The authors acknowledge the help of Associate Professor Dr. Azmi Mohd Tamil and Dr. Azmawati Mohammed Nawi from the Community Health Department for their statistical input.</div>\r\n', 1),
(67667, 67653, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	Rheumatoid Arthritis (RA) is the most common autoimmune inflammatory polyarthritis (Rothschild et al. 1988). It is characterized by synovial inflammation that may lead to irreversible destruction of the cartilage, tendons and bones. RA is also associated with autoantibody production (rheumatoid factor and anti&ndash;citrullinated protein antibody (ACPA) with systemic features which may involve the cardiovascular, pulmonary and haematological systems (McInnes &amp; Schett 2011). The consequent joint damage in RA often leads to deterioration in quality of life and severe functional impairment (Welsing et al. 2001).</div>\r\n<div>\r\n	Despite the major advances in the treatment of RA with novel biologic therapies, there are still several unmet needs. These include reliable biomarkers which are able to accurately reflect the disease activity, prognosis and severity of the disease. Serum C-reactive protein (CRP) and Eryhtrocyte Sedimentation Rate (ESR) are the classical inflammatory biomarkers that are being used in the disease activity assessment of RA. Unfortunately, these biomarkers are non-specific and can be raised in many other inflammatory conditions eg infections and malignancies. A specific biomarker has a pivotal role in the management of RA to avoid undertreatment which leads to joint damage, or overtreatment which may pose unnecessary risks.</div>\r\n<div>\r\n	Matrix metalloproteinase-3 (MMP-3) is a proteolytic enzyme and it is produced mainly by the chondrocytes and synovial fibroblasts. It has been found to be elevated in the serum and synovial fluid in patients with RA (Kobayashi et al. 2007; Mahmoud et al. 2005; Ribbens et al. 2002). Its production is upregulated by the proinflammatory cytokines IL-1&beta;, TNF, IFN&gamma;, and IL-17A, as well as by serum amyloid A (SAA) in RA (Galliera, et al. 2010; Hueber et al. 2010). It also contributes to the progression of RA by promoting the recruitment of many other inflammatory cells ie neutrophils, monocytes and T cells (Jones et al. 2008). Since MMP-3 is produced in the inflamed joint, and subsequently circulated in the blood stream, it is a potential biomarker which may indicate the disease activity in RA (Zucker et al. 1994). In addition, elevated levels of MMP-3 may indicate ongoing destructive processes within the bones and synovium with resultant joint damage. The above however, is theoretical and has to be supported by clinical studies.</div>\r\n<div>\r\n	Therefore, the main aim of the present study was to explore the correlation of serum MMP-3 with disease activity, joint damage and functional disability in RA.</div>\r\n<div>\r\n	materials and METHODS</div>\r\n<div>\r\n	Study Design and Study Population</div>\r\n<div>\r\n	This was an observational, case-control study involving RA patients who were under follow-up at the Universiti Kebangsaan Malaysia Medical Centre. This study was approved by the institutional review board and ethics committee (DLP 2013-039). The following were the inclusion criteria of this study; i) Patients diagnosed with RA based on American College of Rheumatology 1987 (Arnett et al. 1988) or 2010 (Aletaha et al. 2010), ii) Patients aged 18 yrs and above.</div>\r\n<div>\r\n	The following patients were excluded: i) Patients who had previous hand and/or wrist surgeries which may obscure the assessment of the hand radiographs, ii) Patients with active infections, iii) Patients with malignancies.&nbsp;</div>\r\n<div>\r\n	The subjects with RA were assessed for DAS28 (disease activity score based on 28 joints) and Stanford Health Assessment Questionnaire (HAQ)8-item Disability Index (HAQ-DI). A total of 77 RA patients and 18 healthy controls consented and were tested for serum matrix metalloproteinase-3 (MMP-3). The hand radiographs of the RA patients were scored using the Modified Sharp Score (MSS) by a single musculoskeletal radiologist who was blinded to the subjects.&nbsp;</div>\r\n<div>\r\n	Data Collection</div>\r\n<div>\r\n	The medical records of all the subjects with RA were reviewed to gather information on the disease duration and seropositivity. The sociodemographic data, Patient Global Assessment (PGA) scores and Stanford HAQ-DI scores were determined by questioning the subjects. The Stanford HAQ &ndash;DI consists of eight questions regarding the functional limitations in performing their activities of daily living. Patients were required to answer &nbsp;on how difficult it was to perform an activity on a scale of 0 (without any difficulty) to 3 (unable to do). The scores were averaged to a single total score (Fries et al. 1980). The Patient Global Assessment (PGA) required patients to state how they felt generally using a scale of 0 (very well) to 10 (very poor) (Rohekar &amp; Pope 2009). &nbsp;</div>\r\n<div>\r\n	In this study, the DAS 28 calculation was performed by certified rheumatologists. The four parameters used to calculate &nbsp;DAS 28 were; number of swollen joints, number of tender joints, C-reactive protein (CRP) or erythrocyte sedimentation rate (ESR), and the patient&rsquo;s global assessment (Fransen &amp; van Riel 2005; Prevoo et al. 1995). A DAS 28 of 3.2 and above (moderate to high disease activity) is considered as active disease.&nbsp;</div>\r\n<div>\r\n	Using the modified Sharp scoring (MSS) system (Pincus et al. 1997), each joint was scored for joint space narrowing and erosion. Fifteen sites in each hand and wrist were examined for joint space narrowing on a scale of 0-4. Similarly, the erosions were scored individually at 16 sites in each hand and wrist.</div>\r\n<div>\r\n	Serum Matrix Metalloproteinase-3(MMP-3)</div>\r\n<div>\r\n	Serum samples from all subjects were stored at &minus;80˚C until analysis. Serum levels of soluble MMP-3 were measured with a human MMP-3 sandwich enzyme immunoassay detection kit (Quantikine, R&amp;D systems) according to the manufacturer&rsquo;s instructions. Measurements were done in duplicate. Serum samples were placed in designated microwells and both positive and negative calibrators were added to the designated microwells in order to determine a standard curve. The plates were then incubated for 30 mins at 26˚C and washed with a buffer solution.&nbsp;</div>\r\n<div>\r\n	Statistical Analysis</div>\r\n<div>\r\n	The statistical analyses were carried out using the Statistical Package for Social Sciences (SPSS) package for Windows version 21. Continuous variables were expressed as mean &plusmn;SD. Differences between the RA patients and healthy controls were analysed using the Chi-square test (categorical variables) and independent t-test (continuous variables). Correlations between the serum MMP-3 levels and clinical indicators were analysed by the Spearman&rsquo;s rank order correlation test. A p value of &lt;0.05 was considered statistically significant.</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	Demographic data&nbsp;</div>\r\n<div>\r\n	A total of 77 RA patients and 18 healthy controls were enrolled in this study. The majority of the patients were females (89.6%). The subjects were predominantly Malays for both the RA patients (53.3%) and the controls (88.9%). The mean age of the subjects with RA was 58.6 &plusmn;&plusmn;10.3 yrs whereas for the controls was 56.4 &plusmn; 7.3 yrs. The demographic characteristics were matched between the RA patients and the controls (Table 1). The serum MMP-3 levels were significantly higher among the RA patients (p&lt;0.05). The mean serum MMP-3 level in RA patients was higher than the normal range (18&ndash;60 ng/mL) whereas the healthy controls were within the range. &nbsp;</div>\r\n<div>\r\n	Correlations between serum MMP-3 levels and clinical measures of disease activity, joint damage and functional disability</div>\r\n<div>\r\n	Spearman&rsquo;s rank order correlation test showed significant correlations between the serum MMP-3 levels and MSS (r value of 0.327) &nbsp;and HAQ-DI (r value of &nbsp;0.256) (Table 2). The strength of the relationship however was only moderate for MSS (r within 0.3-0.5) &nbsp;and weak for the HAQ-DI (r within 0.1-0.3). The mean serum MMP-3 levels in RA patients with radiographic joint erosions was significantly higher (p=0.017) than in patients without erosions (Figure 1). Likewise, the subjects with significant functional impairment i.e HAQ-DI &ge;1; had significantly higher mean MMP-3 levels (90.7 ng/ml) compared to RA patients without significant disability (80.3 ng/ml). There was no appreciable difference in this regard (p=0.268) between the active disease and inactive disease groups (Figure 1). Age and disease duration had an inverse relationship with the serum MMP-3 levels. There were no significant correlations with the disease activity and inflammatory markers such as ESR and CRP (Table 2).</div>\r\n<div>\r\n	A multiple regression analysis was performed to predict serum MMP-3 from HAQ-DI, MSS, DAS 28, ESR and CRP. These variables statistically significantly predicted serum MMP-3, F(5,95) = 7.368, p &lt; 0.005, R2 = 0.450. However, only HAQ-DI added significantly to the prediction, p&lt;0.05.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	This study highlighted that serum MMP-3 levels had a significant association with radiographic joint damage and functional impairment in RA. There is paucity of data on the predictive role of serum MMP-3 with regard to functional disability. However, a Japanese study (Shinozaki et al. 2007) reported that elevated serum MMP-3 predicted progressive disability based on JHAQ (Japanese version of HAQ) especially in female patients without corticosteroid. Galil et al. (2016), on the contrary, disclosed no positive correlations between serum MMP-3 levels and HAQ. Of note, the patients recruited by Galil et al. (2016) had joint symptoms for less than a year and hence, did not have significant functional disability. Our patients, on the other hand, had a mean disease duration of 10.5 yrs. We found that HAQ-DI was the only variable which significantly predicted serum MMP-3 using multiple regression analysis.</div>\r\n<div>\r\n	The finding of significant positive correlation between radiographic joint damage and serum MMP-3 echoes the results of the previous studies in this regard (Cheung et al. 2000; Cunnane et al. 2001; Galil et al. 2016). Some of these studies could demonstrate the above link despite using a different joint scoring system i.e the Larsen score (Yamanaka et al. 2000). MMP-3 acts directly on the matrix of the cartilage. In animal models, MMP-3 was inducible and detectable in chondrocytes even in the early phase of arthritis (Okada et al. 1989; Singer et al. 1995). &nbsp;Progressive degradation of the cartilage leads to joint space narrowing. Articular erosions are due to bone resorption which is facilitated by MMP-3-mediated removal of the outer osteoid layer (Cunnane et al. 2001) .</div>\r\n<div>\r\n	In contrast to many previous studies (Ally et al. 2013; Galil et al. 2016; Ichikawa et al. 1998), we found no relationship between serum MMP-3 and either disease activity or inflammatory markers (ESR,CRP). The difference in the study population may partially explain our discrepant findings. We recruited all RA patients regardless of disease duration. Many of the studies which demonstrated a link between disease activity and MMP-3 included patients with only early disease (Urata et al. 2012; Ally et al. 2013). The mean DAS 28 in this study was 2.19 (in remission). In general, patients with early disease tend to have higher disease activity due to unoptimised treatment and while in the interim before the onset of action of disease modifying anti-rheumatic drugs.</div>\r\n<div>\r\n	We do acknowledge the limitations of our study. As with most cytokines and biomarkers, MMP-3 levels may fluctuate in the blood circulation. Ideally, 2-3 samples of blood should have been obtained from each subject on separate occasions. The average measurement of MMP-3 could have been used in the analysis. Each subject only had a single blood sample of MMP-3 taken; for convenience. Besides, ultrasound has been proven to be more sensitive in detecting joint erosions in RA. Early or mild erosions can be easily overlooked with plain radiographs (Filippucci et al. 2006).</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	In conclusion, serum MMP-3 is a useful predictor of radiographic joint damage and functional disability in RA. The findings of this study have clinical implications. Novel therapies in RA should target MMP-3 to minimise joint destruction. Several models of MMP-3 inhibitors are currently under phase I and II of clinical trials.</div>\r\n', 1);
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(67674, 67660, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Cardiovascular disease (CVD) is still the most common cause of mortality worldwide (Santulli 2013). This may be attributed to the increase in the prevalence of CVD risk factors such as hypertension, dyslipidemia, smoking and obesity (Berry et al. 2012; Santulli 2013). Among the young population, lack of health screening may also predispose to increase CVD prevalence and they may present with advanced stage of the disease (Kuklina et al. 2010). Thus, there is a requirement to develop a screening method that is simple, non-invasive, reproducible and less time consuming.</div>\r\n<div>\r\n	The assessment of vascular function via finger photoplethysmography (PPG) has gained popularity since this method has the characteristics mentioned above at significantly lower cost. PPG detects blood volume changes of the finger arterial bed and is influenced by several factors such as ventricular ejection, aortic and peripheral arterial stiffness (AS), peripheral arterial resistance and blood viscosity (Elgendi 2012; Millasseau et al. 2002). Several common parameters that were derived from finger PPG are stiffness index (SI), reflection index (RI), crest time and a, b, c, d and e waves, and all were proposed to be indices of vascular function and vascular ageing (Millasseau et al. 2002; Brillante et al. 2008; Baek et al. 2007; Hashimoto et al. 2005; Takazawa et al. 1998; Tsai et al. 2007).</div>\r\n<div>\r\n	Recently, Chellappan et al. (2008) has introduced another parameter designated as PPG fitness index (PPGF). PPGF was derived from AC component of PPG morphological changes against a healthy 19-yr-old reference (gender specific) identified from study population as seen in Figure 1 (Chellappan et al. 2008). PPGF also has a good reproducibility record (Chellappan 2010). Compared to the previous PPG waveforms parameters, PPGF was derived by analyzing the entire waveforms and not concentrating on a specific part; e.g. systolic or diastolic component. The potential of derived PPGF was further improved through empirical modeling to produce a vascular health indicator named vascular risk prediction index (VRPI) (Chellappan 2009). An aged based empirical model was established among Malaysian population, which has been named as Model 1 for age group between 19 to 44 and Model 2 for 45 to 66 yrs. Hypertension, diabetes mellitus, hypercholesterolemia, obesity and smoking are the CVD risk factors considered in this model development and were compared with the healthy subjects. VRPI classified the subject into very low risk, low risk, moderate risk or high risk to have CVD risk factor (Chellappan 2009). VPRI was derived from the regression analysis, the difference between the observed PPGF value and the predicted PPGF value which is called the residual [risk score (e)]. In this study, we have selected Model 1 to be tested among young men with CVD risk factor from Malaysian population (Chellappan 2009) (Table 1).</div>\r\n<div>\r\n	CVD is closely related with atherosclerosis. Atherosclerosis involves inflammatory reactions in every step (Stoger et al. 2012; Drechsler et al. 2010). In advanced lesion, there is development of fibrous tissue and calcification and these structural changes may lead to functional disability which can be measured as arterial stiffness (Crowther 2005; Zieman et al. 2005). Based on this knowledge, several established markers of CVD were found such as C-reactive protein (CRP), carotid intima media thickness (CIMT), pulse wave velocity (PWV) and augmentation index (AI) (Anderson et al. 2009; Polak et al. 2011; Kaptoge et al. 2010). CRP is an inflammatory marker, PWV and AI are markers of arterial stiffness/vascular function and CIMT is an atherosclerotic marker. However, all these markers either need an invasive procedure (needle prick), experience technician (CIMT, PWV) or expensive device (CIMT, PWV, AI). These factors may limit the screening process among the subjects especially in an open and large scale setting.</div>\r\n<div>\r\n	Chellappan et al. (2008) proposed that PPGF and VRPI can be used as assessment of vascular health to detect vascular dysfunction. However, further validation is important before PPGF and VRPI can be incorporated as vascular health screening tools. Therefore, this study assessed the correlations between PPGF and other CVD risk markers namely CRP, CIMT, PWV and AI. This study also aimed to investigate the independent variables of PPGF which are still unknown, but theoretically related to ventricular ejection and aortic stiffness. Lastly, this study compared the PPGF among subjects with and without CVD risk factors and assessed the sensitivity of VRPI in predicting subjects with CVD risk factors.</div>\r\n<div>\r\n	materials and Methods</div>\r\n<div>\r\n	Ethical approval was obtained from the Ethics Committee of Universiti Kebangsaan Malaysia Medical Centre (Project Code: FF-262-2011). &nbsp;Subjects were recruited around Klang Valley which is an urban and the most densely populated area in Malaysia. The recruitments were made through public health screening. Subjects were men aged between 20 to 40 yrs, who were healthy or had any CVD risk factor. CVD risk factors include systolic or diastolic blood pressure (BP) 140/90 mmHg or on antihypertensive medication, abdominal obesity (waist circumference &gt; 90 cm), smoker, dyslipidemia (Total cholesterol &gt; 6.2 mmol/L or low density lipoprotein &gt; 4.1 mmol/L or triglyceride &gt; 1.7 mmol/L or high density lipoprotein &lt; 1.02 mmol/L) or family history of premature CVD (Aminuddin et al. 2014). These risk factors may affect peripheral and central vascular functions and cause vascular damage, that may be detected by PPGF. Exclusion criteria were: CVD, diabetes mellitus and any chronic inflammatory diseases. These factors were excluded since they are associated with advanced vascular damage and contradict with the focus of this study which was screening for early vascular lesion. Sample size was 24 subjects per group as calculated by using a published formula based on previous paper on PPGF (Eng 2003; Chellappan et al. 2008). Subjects were divided into three groups, healthy subjects (HG), subjects with one risk factor (G1) and subjects with at least two risk factors (G2) and this was done to determine whether PPGF has the ability to differentiate these groups.</div>\r\n<div>\r\n	Ethics, consent and permissions</div>\r\n<div>\r\n	All the subjects gave written informed consent for their participation.</div>\r\n<div>\r\n	Measurement of body weight, height and waist circumference</div>\r\n<div>\r\n	On the day of the procedure, the subjects were asked to fast and to avoid smoking for at least six hrs. Weight was measured by using digital scale (SECA, German). Height was measured by using a wall-mounted stadiometer (SECA, German) with the subjects not wearing shoes. Body mass index (BMI) was then calculated by dividing weight with height (kg/m2). Waist circumference (WC) was measured by using a measuring tape over the bare midriff midway between the lowest rib and iliac crest at the end of normal expiration.</div>\r\n<div>\r\n	Measurement of blood parameters</div>\r\n<div>\r\n	Blood was taken from the antecubital vein after fasting for at least eight hrs. Samples were sent to Gribbles Pathology Laboratory, Petaling Jaya for estimation of total cholesterol (TC), low density lipoprotein (LDL), high density lipoprotein (HDL), triglyceride (TG), high sensitivity C reactive protein (hs-CRP) and fasting blood sugar (FBS). The details of the method for the blood test was published earlier (Aminuddin et al. 2014).</div>\r\n<div>\r\n	Measurement of central blood pressure (BP), augmentation pressure, augmentation index and forward pressure</div>\r\n<div>\r\n	Subjects were asked to lay supine. A blood pressure cuff (Vicorder system, SMT Medical, Wuerzburg, Germany) was put on the right arm of the subject for measurements of the brachial systolic and diastolic BP (bSBP/bDBP) via oscillometric method. The system also recorded the brachial pressure waveforms which were then transformed to the aortic pressure waveforms by using a brachial-to-aortic generalized transfer function and calibrated to brachial mean BP and DBP for the estimation of central SBP (cSBP), DBP (cDBP) and central pulse pressure (cPP). The estimated aortic pressure waveform was also used to measure augmentation pressure (AP) and forward wave pressure (P1) (Figure 2) (Aminuddin 2015). AI was measured as [(augmentation pressure (AP) /pulse pressure x 100] (Laurent et al. 2007).</div>\r\n<div>\r\n	Measurement of PWVCF</div>\r\n<div>\r\n	This measurement was done using Vicorder system (SMT Medical, Wuerzburg, Germany). The subjects were asked to lie supine with head propped up at 45&deg;. A neck cuff was placed around the neck at the level of common carotid area, while a thigh cuff was placed at the upper thigh. The distance between the suprasternal notch and mid-thigh cuff was determined by using a measuring tape (D). The time taken for the pressure wave to travel from the aorta to the femoral artery was measured from the foot to foot of the pressure wave (T) by the system. &nbsp;PWVCF was then calculated by the system as D over T (m/s).&nbsp;</div>\r\n<div>\r\n	Measurement of PPGF</div>\r\n<div>\r\n	The measurement was done in a room with controlled temperature between 20-25&deg;C. The subject was in supine position. After resting for five mins, the PPG probe was attached to the left index finger of the subject and measurement was done for 120 secs. The signals were acquired through the serial port of pulse-oximeter modules (NiVaRiX 1.0, Universiti Kebangsaan Malaysia). The system was connected to a personal computer running the application developed by the manufacturer of the NiVaRiX 1.0. The sampling rate was 100 Hz and the resolution was 16 bits. PPGF was derived by using real-time PPG signal with age as a confounding factor since age is a non-modifiable risk factor of CVD. The application also provided an estimation of VRPI. VRPI consisted of very low risk, low risk, moderate risk and high risk, which refer to the risk of having any risk factor of CVD (Chellappan 2009).</div>\r\n<div>\r\n	Measurement of CIMT</div>\r\n<div>\r\n	Measurement was done with subjects lying supine with head supported with one pillow in a dimmed-light room. CIMT was defined as the distance between lumen-intima interface and media-adventitia interface. A guideline for measurement of CIMT published by American Society of Echocardiography was adopted (Stein et al. 2008). Intima-media thickness of the right and left common carotid arteries (CCA) were measured using B mode ultrasound (Vivid-I) via linear array probe (10 MHz). The CIMT measurement was done with the subject&rsquo;s head lifted upwards and turned slightly to the contralateral side. CIMT measurement was done on the smallest vessel diameter (diastole) by using a caliper on the far wall of the CCA. Three measurements were taken within one cm proximal to the carotid bulb in the right (RCIMT) and left CCA (LCIMT) and the average were calculated on each side. All measurements were done by a single trained technician.&nbsp;</div>\r\n<div>\r\n	Statistical analysis</div>\r\n<div>\r\n	The normality of the data was checked using Kolmogorov-Smirnov test. &nbsp;All the data were presented as mean&plusmn;SD, except for hs-CRP which was in median&plusmn;interquartile range as it was not normally distributed. Hs-CRP value was also log transformed and was used in further analysis. Data were analyzed via SPSS version 15. The differences between groups were compared by ANOVA, and when adjustment for confounders were needed then ANCOVA was used. Age and race were important confounders for all the vascular properties. In addition, based on previous studies, heart rate was important confounder for PWV and AI (Tomiyama et al. 2010; Laurent et al. 2007) and height was important confounder for AI (Laurent et al. 2007). Post-hoc Gabriel analysis was done for parameters that were significant between groups (p&lt;0.05). Associations between the markers were determined by Pearson correlation r. Multiple linear regression was used to determine the independent variables of PPGF.</div>\r\n<div>\r\n	Sensitivity and specificity analysis</div>\r\n<div>\r\n	The sensitivity of VRPI to detect those with CVD risk factor was calculated as [true positive (TP)/ (TP + false negative)] x 100% and the specificity was calculated as [true negative (TN)/ (TN + false positive)] x 100%. In the present study, positive result refers to those having VRPI at either low risk, moderate risk or high risk and negative result refers to those having VRPI at very low risk. &nbsp;As this study is focused on young subjects, low risk is also considered as a positive result. True positive were those with CVD risk factor and VRPI results were low/moderate/high risk. True negative refers to those without CVD risk factor and VRPI result was very low risk. False positive refers to those without CVD risk factor but VRPI results were low risk/moderate risk/high risk. False positive refers to those with CVD risk factor but VRPI results were very low risk (Table 2).&nbsp;</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Table 3 summarized the general characteristics of the subjects. A total of 114 subjects were involved. The mean age of all the subjects was 28.94 &plusmn; 4.86 yrs. There were significant increasing trends for age, bSBP, bDBP, brachial mean arterial pressure (bMAP), cSBP, cDBP, WC, BMI, hs-CRP and lipid profiles (except for HDL which had significant reducing trend) as the number of risk factors increased (P&lt;0.05 for all).</div>\r\n<div>\r\n	Table 4 summarized the vascular properties of the groups. &nbsp;Post-hoc analysis revealed that the level of AI was significantly increased in G2 when compared to G1 and HG, which remained significant after adjustment for the age, race, HR and height. No differences were observed in terms of PWVCF, AP, P1, RCIMT, LCIMT and PPGF between groups.&nbsp;</div>\r\n<div>\r\n	Table 5 showed the correlations between PPGF with other vascular properties and risk factors. PPGF was significantly correlated with height, DBP, MAP and PWVCF. In multiple linear regression, the independent variables of PPGF were P1 (Beta = 0.35, p &lt; 0.01), PWVCF (Beta = -0.26, p &lt; 0.01), SBP (Beta = -0.26, p = 0.04) and height (Beta = 0.24, p &lt; 0.01).</div>\r\n<div>\r\n	For sensitivity study, the analysis showed that the sensitivity of VRPI to identify young subjects with CVD risk factor was 82.02% and the specificity was 15.38%. This indicated that VRPI had high sensitivity but low specificity in detecting those with CVD risk factors.&nbsp;</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The use of peripheral vascular signal such as via PPG has gained a lot of focus since this method is easy, reproducible, non-invasive and cost-friendly. PPGF is developed by comparing PPG signal of a subject with a healthy young subject. The value of PPGF is in percentage and the higher value indicates better vascular health. In the present study, the relationship between PPGF and other CVD biomarkers were studied. This study found that PPGF correlated independently with PWVCF, a gold standard measurement of aortic stiffness (AS). This suggests that AS is one of the determinants of PPGF and PPGF may be a potential marker of AS.&nbsp;</div>\r\n<div>\r\n	Past research that studied the correlations between other PPG waveforms such as stiffness index (SI) digital volume pulse (SIDVP) and B/A ratio with aortic stiffness indices also found similar finding, but at different strengths of correlation. Study by Salvi et al. (2008) found that among subjects with risk factors and CVD, SIDVP correlated to PWV (r=0.55). Among the healthy subjects, Millasseau et al. (2002) found that SIDVP correlated to PWV (r=0.65). Another study by Chen et al. (2005) observed that among hypertensive subjects, SIDVP correlated to aortic SI (r=0.31) and distensibility (r=-0.34) measured via echocardiogram. Study by Woodman et al. (2003) found that SIDVP correlated to PWV (r=0.54). Hashimoto et al. (2002) observed that among hypertensive subjects, PWV correlated to B/A (b/a) ratio obtained by analyzing the second derivative PPG, which is a proposed index of AS. However, this correlation became insignificant after adjustment for the age and MAP (r=0.16). A study by von Wowern et al. (2015) found that b/a ratio was correlated significantly with PWV (r=0.56). Compared to above studies, PPGF correlation with PWV was weak (r=-0.27) which suggest that PWV may not be the only marker that influence PPGF. We found that forward pressure (P1), SBP and height also influenced PPGF. Again, all these factors contributed only about 18% variability of PPGF. Other factors that affected PPGF were still unknown and should be investigated further.&nbsp;</div>\r\n<div>\r\n	In the present study, no correlation was found between PPGF and age. This can be due to low sample size and narrow range of age. Furthermore, the influence of aging on the PWVCF as the determinant of PPGF may not be apparent among the young subjects (Aminuddin et al. 2014, McEniery et al. 2005). In contrast, study by Chellappan et al. (2008) found significant decrease in PPGF as the age increased. In her study, the age range was wider which was from 19 to 68 yrs.</div>\r\n<div>\r\n	In the present study, no correlation was found between PPGF and CIMT. Previous study by Wykretowicz et al. (2009) found that SIDVP was not correlated to CIMT. Another study found that among Japanese subjects there was a significant correlation between b/a ratio with CIMT but this correlation was weak (&beta;=0.069) (Tabara et al. 2016). This suggests that CIMT and PPGF reflects different vascular characteristic and should be used complimentary with each other and not interchangeably. This study also found no correlation between PPGF and hs-CRP. Similar finding was observed in a study by Kawada &amp; Otsuka (2013) which found no correlation between b/a ratio and CRP. In contrast, study by Tsai et al. (2007) observed that SIDVP was correlated with CRP but weak (r=-0.17). Their subjects&rsquo; was much older (mean age=41 yrs) and only involved hypertensive subjects, which may account for the discrepancy in the results.</div>\r\n<div>\r\n	The results showed that there was no significant difference in PPGF among the study groups. This was unexpected since previous study found that PPGF was significantly reduced among young and old subjects with CVD risk factors (Chellappan 2009). There are several reasons for this. Firstly, previous study involved subjects who had more severe clinical characteristic compared to the current study and the cut-off point to be in the risk factors groups were much higher (Chellappan 2009). For example, hypertension was diagnosed if DBP&gt; 100 mmHg compare to the current study which was &gt;140/90 mmHg. Low HDL was diagnosed as HDL&lt;0.85 mmol/L compare to the current study which was &lt;1.02 mmol/L. They also involved diabetic subjects, which usually had vascular damage by the time of diagnosis but in the current study diabetic subjects were excluded. Thus, their subjects may have had significant vascular damage that can be detected as low PPGF. In addition, the mean value of physical and biochemical status (SBP, DBP, TC, TG, LDL) of the subjects with at least two risk factors in the present study were still within normal range. Secondly, PPGF was influenced by PWV. No difference in PWV between the groups in the current study might also explain the insignificant difference for PPGF between the groups. No difference observed for AS measured by PWVCF between the groups were in line with previous studies in young subjects (Dart et al. 1991; Ferreira et al. 2007; Toikka et al. 1999). This can be due to the fact that our subjects were still young and aortic stiffening may not be significant at the early stage of the occurrence of the risk factor (Aminuddin et al. 2014).</div>\r\n<div>\r\n	The significantly increased AI among those with risk factors was in line with previous studies (Jatoi et al. 2007; Kyvelou et al. 2010; Urbina et al. 2012; Wilkinson et al. 2000). Previous studies also suggested that AI may be a sensitive index of vascular dysfunction among the young subjects (Aminuddin et al. 2014; McEniery et al. 2005; McEniery et al. 2010). Increased AI may be due to increased ventricular ejection, aortic stiffness or total peripheral resistance (Casey et al. 2012). The present finding suggests that AI can be used as an early marker of vascular damage due to CVD risk factor.</div>\r\n<div>\r\n	The novel finding of the present study was that the sensitivity of VRPI to identify young subjects with CVD risk factor was nearly 82%, which was quite high. However, the specificity was low (nearly 15%). High sensitivity result means that the test rarely misdiagnosed those with positive test, and those with negative test can be said as having high probability to not have the disease. In other words, high sensitivity test is beneficial to exclude a disease. Thus, for VRPI, it can be said that those with negative result (very low risk) is safe and they can repeat the measurement after one year for continuous assessment of vascular health. It is also suggested that those with positive result (low risk, moderate risk and high risk) should be referred for further evaluation of CVD risk factor such as measurement of BP and lipid profiles. In this context, VRPI can be used as a first hand screening for CVD risk factor. VRPI can be also used to assess the changes in vascular health due to lifestyle intervention such as dietary modification and exercise program.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	In conclusion, PPGF is independently associated with PWV and may be a potential marker of AS, which may need further validation. VRPI can also be used as early screening of CVD risk factors since the method is sensitive.&nbsp;</div>\r\n<div>\r\n	Acknowledgement&nbsp;</div>\r\n<div>\r\n	The present study was supported by Pusat Perubatan Universiti Kebangsaan Malaysia (PPUKM) Fundamental Grant (FF-262-2011) and Universiti Kebangsaan Malaysia grant (GGPM-2011-078).</div>\r\n', 1),
(67680, 67666, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Lead (Pb) poisoning is one of the major public health concerns worldwide due to its adverse effects to the human body systems (Iyer et al. &nbsp;2015). Lead poisoning can occur as a result of exposure to lead through air, household dust, sand, water and commercial productssuch as paint (Cao et al. 2014). Risk factors of lead poisoning include the use of leaded gasoline, occupational exposure to heavy metals and battery recycling, water harvesting techniques using old pipes as well as the level of education among parents, guardian or child&rsquo;s primary caregiver (Brown et al. 2005; Albalak et al. 2003). Lead poisoning in human is indicated when blood lead levels reach &ge;&ge; 10 &mu;g/dL whole blood (Taylor et al. 2013). The fatal dose is estimated at 500 mg of absorbed lead (Noji &amp; Kelen 1989). The threshold for toxic effects of lead is yet to be identified. However, blood lead levels of &ge;&ge; 80 &mu;g/dL in children were reported to cause encephalopathy (Cao et al. 2014), whereas a lower blood lead level caused adverse effects on the central nervous system, though symptomless (Iyer et al. 2015).</div>\r\n<div>\r\n	High blood lead levels were reported to affect physical development, auditory system, kidneys, bones and the most feared was the effect on the nervous system. Lead was reported to be highly neurotoxic and particularly affected the developing central nervous system (CNS) (Antonio-Garc&iacute;a &amp; Mass&oacute;-Gonzalez 2008). Previous studies of lead toxicity on the nervous system of mice found disruption in cognitive function due to changes in the cell morphology in certain regions of the brain (Huang &amp; Schneider 2004; Yang et al. &nbsp;2003).</div>\r\n<div>\r\n	The present study hypothesized that lead-induced brain toxicity is highly attributable to oxidative stress. There is increase in evidence that chronic lead exposure plays an important role in the disruption of prooxidant: antioxidant balance in the brain tissue and alters brain histology which leads to physiological dysfunction (Bokara et al. 2008; Hamed et al. 2010). Oxidative stress occurs in the plasma and brain tissue in lead-exposed rats as evidenced by decreased important intracellular antioxidant enzymes and increased lipid peroxidation products (Xia et al. 2010). To the best of our knowledge, past studies have observed the effect of lead consumption along with chelating agent or other supplementary elements. Other researchers have observed the histopathological changes in the lead exposed group (without treatment).Our interest in the present study was to evaluate the brain histopathological and biochemical effects after the consumption of lead, along with chelating agent from Malaysian palm, tocotrienol-rich fraction (TRF) treatment.TRF is a well known therapeutic agent as it possesses several medicinal properties including antioxidant, anti-inflmmatory and effective free radical scavenging agent (Abd Aziz et al. 2012).</div>\r\n<div>\r\n	Results obtained from the pilot experiment of the present study found that thirty days of lead ingestion even in a small dose of 0.2% v/w was capable of causing histopathological changes in the hippocampus region of rats (Abd Aziz et al. 2012). The present study was therefore aimed to determine the possible effects of TRF on rat brain exposed to 0.2% v/w lead. Determination of lead, TRF, superoxide dismutase (SOD) activity and malondialdehyde (MDA) levels as well as microscopic observation of brain tissue in the hippocampus were performed in order to better understand the involvement of oxidative stress as a mechanism of neurotoxicity oflead.</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	Animals</div>\r\n<div>\r\n	Ethical approval was obtained from the Universiti Kebangsaan Malaysia Animal Ethics Committee (UKMAEC), with approval number of PP/ANAT/2011/FAIRUZ/19-MAY/377-MAY-2011-MAY-2012 prior to the commencement of study. A total of eighteen male Sprague Dawley rats (6-weeks old, weighing 150-200 gm) were obtained from the Laboratory Animal Resource Unit (LARU), Universiti Kebangsaan Malaysia. The rats were housed one animal per cage, acclimatized for one week, fed with commercial rat chow (Gold Coin, Malaysia) with distilled water ad libitum and maintained under controlled environment of 12 hrs light and dark cycle.&nbsp;</div>\r\n<div>\r\n	Experimental Design</div>\r\n<div>\r\n	Rats were randomly divided into three groups, with each group comprising of six animals (n=6). The groups were i) control group which received distilled water ii) group which received lead acetate (0.2% w/v) in drinking water, and iii) group which received lead acetate (0.2% w/v) in drinking water and daily force-fed with tocotrienol-rich fraction, TRF (Golden Hope Bioganic Sdn. Bhd., Malaysia) using oral gavage at 200 mg/kg body weight. TRF used in the study composed of 235.8 mg/g (31.16%) of &gamma;-tocotrienol (GTT), 111.5 mg/g (14.74%) of &delta;&delta;-tocotrienol (DTT), 208.5 mg/g (27.56%) of &alpha;-tocotrienol (ATT), 168.9 mg/g (22.33%) of &alpha;&alpha;-tocoferol (ATF) and 31.8 mg/g (4.21%) of &beta;-tocotrienol (BTT). In all groups, rats were maintained for thirty days and were sacrificed at the end of exposure. Histopathological study of the hippocampus was done using light microscope following hematoxylin and eosin (H&amp;E) staining (Merck, Germany). In addition, another brain hemisphere were homogenized with ice-cold buffer (Tris-HCL, pH 7.5) (Sigma, United States of America) and centrifuged at 10,000 x g at 4˚C for 10 mins. Brain lead levels were determined using atomic absorption spectroscopy (Perkin Elmer, United States of America), brain TRF levels by high-performance liquid chromatography (Shimadzu Corporation, Japan), erythrocyte superoxide dismutase (SOD) enzyme activity by antioxidant enzyme assay (Cayman, United States of America) and brain malondialdehyde (MDA) levels by lipid peroxidation product assay (Cayman, United States of America).</div>\r\n<div>\r\n	Brain Histological Study</div>\r\n<div>\r\n	A hemisection of brain was analyzed for histopathological changes within the hippocampal subregions following lead exposure. Brain tissues were processed according to the standard protocol for light microscopy (Olympus, Japan). Tissues were fixed with 10% formalin (Ajax, Australia), dehydrated through graded alcohol series (25%-100%) (Hayman Ltd., England), cleared in toluene (Systerm Chemicals, Malaysia) and embedded in paraffin wax (Fisher Scientific, United States of America). Serial sections of 3 &mu;m thickness were made using a microtome (Leica, Germany) and stained with H&amp;E staining (Merck, Germany). Quantitative estimation of the number of apoptotic-like cells was done using an image analyzer (Leica, Germany) at x 20 objective based on a described method (Haynes et al. 2001).</div>\r\n<div>\r\n	Atomic Absorption Spectroscopy</div>\r\n<div>\r\n	The brain lead concentration was analyzed by atomic absorption spectrophotometer (Perkin-Elmer, United States of America) at 283.3 nm based on a described method with slight modifications (Hamed et al. 2010). Brain hemisphere was weighed (1 gm) and placed into a polypropylene tube. Nitric acid, HNO3 (Merck, Germany) (1ml) was added to the brain tissue and digested in a shaking bath (Memmert, Germany) at 60˚C for 30 mins. Supernatant (100 &micro;l) was then taken from the clear solution and diluted (1:5 v/v) with deionized water. The standard curve was obtained by adding lead standard solutions (Fisher Scientific, United States of America) with increase in concentration corresponding to 20, 40, 60, 80 and 100 &micro;g/dL lead (Fisher Scientific, United States of America). Brain lead values were expressed as &micro;g/gm brain tissue.</div>\r\n<div>\r\n	High-Performance Liquid Chromatography</div>\r\n<div>\r\n	The brain TRF levels were analysed by reverse-phase high performance liquid chromatography with fluorescent detector of EX 294; EM 330 nm (Shimadzu Corporation, Japan). Brain homogenate (200 &micro;l) were added to 50 &micro;l of butyllated hydroxytoluene (BHT, 10 mg/ml (Merck, Germany) in 95% ethanol (Hayman Ltd., England). Another 1ml of 100% ethanol was then added to the sample and vortexed (Stuart, Germany). Samples were centrifuged (Jouan, France) at 5000 rpm at 18˚C for 15 mins. Hexane (3 ml) (Merck, Germany) was added to the supernatant and vortexed, before centrifuged again at 5000 rpm at 18˚C for 15 mins. The top layer (2.5 &mu;l) was pipetted out (Eppendorf, France) into new tube and dried using vacuum concentrator (Heto Lab, Denmark). Dried samples were added with 100&micro;&mu;l HPLC-grade hexane (Merck, Germany) and filtered using 1 cc/ml, 0.2 &mu;m syringe filter (Sartorius Stedim Biotech, Germany). Filtered samples (30 &mu;l) was further diluted 1:10 with HPLC-grade hexane for analysis. The standard curve was obtained by adding TRF with increasing concentration corresponding to 2, 4, 6, 8, 10 and 14 &mu;g/ml TRF (Golden Hope Bioganic Sdn. Bhd., Malaysia). Brain TRF levels were expressed as &mu;g/ml brain homogenate.</div>\r\n<div>\r\n	Antioxidant Enzyme Assay</div>\r\n<div>\r\n	The total cytosolic and mitochondrial superoxide dismutase (SOD) enzyme activities of erythrocytes in the blood were determined by Cayman Superoxide Dismutase Assay kit (Cayman, United States of America) based on described method (Kanematsu &amp; Asada 1991). Haemolysates were obtained by centrifugation (Jouan, France) of fresh blood samples at 3000 rpm at 4˚C for 10 mins, lysis of erythrocytes with ice-cold distilled water at 1:4 v/v followed by centrifugation at 10000 x g at 4˚C for 10 mins. Prior to analysis, supernatant (haemolysate) was diluted 1:100 (v/v) with sample buffer solution (50 mM Tris-HCL, pH 8.0) (Sigma, United States of America). Samples solution of 10 &mu;l were added to microplate wells filled with 200 &mu;l radical detector assay solution (tetrazolium salt in SOD assay buffer solution, 1:400)(Cayman, United States of America). Reaction was innitiated by the addition of 20 &mu;l xantine oxidase solution (Cayman, United States of America) and the microplate was put on incubated plat shaker (New Brunswick Scientific, United Kingdom) for 20 mins. The standard curve was obtained by adding SOD standard solutions (Cayman, United States of America) with increasing concentration corresponding to 0.025, 0.05, 0.1, 0.15, 0.2 and 0.25 U/ml SOD (Cayman, United States of America). SOD activities were read using ELISA reader (Biotech, United States of America) at 440-460 nm. Erythrocytes SOD activities obtained through the SOD enzyme assay kit (Cayman, United States of America) were compared to the levels of haemoglobin (Hb) concentrations in each haemolysate sample using Cyanomethaemoglobin assay (Sigma, United States of America) based on a described method (Lewis &amp; Shinton 1980). Results were expressed as U/mg Hb.</div>\r\n<div>\r\n	Lipid Peroxidation Product Assay&nbsp;</div>\r\n<div>\r\n	The brain malondialdehyde (MDA) levels were determined by Cayman Malondialdehyde Assay kit (Cayman, United States of America) based on described method (Yagi 1998). Sodium dodecyl sulphate (SDS) solution (100 &mu;l)(Cayman, United States of America) were added to the brain homogenate (100 &mu;l)in a 5 ml tube and shaken thoroughly.Colour reagent (thiobarbituric acid solution) (Cayman, United States of America) (4 ml) were added to the bottom of the tube. Tubes were capped and put vertically in the rack. After all tubes were brought to boil on a heated plate (Leica, Germany) for 1 hr, the chemical reaction was stopped by immediately putting the samples on ice for 10 mins. Samples were centrifuged (Jouan, France) at 1600 x g at 4˚C for 10 mins. Samples (150&micro;&mu;l) were used for each microplate well (Cayman, United States of America). The standard curve was obtained by adding MDA standard solutions (Cayman, United States of America) with increasing concentration corresponding to 0.625, 1.25, 2.5, 5.0, 10.0, 25.0 and 50.0 &mu;M MDA (Cayman, United States of America). Brain MDA levels were determined using ELISA reader (Biotech, United States of America) at 530-540 nm. Results were expressed as &mu;M/ml brain homogenate.</div>\r\n<div>\r\n	Statistical Analysis</div>\r\n<div>\r\n	SPSS software version 19.0 (SPSS Incorporation, United States of America) was used in all analyses. ANOVA, with post-hoc Tukey HSD were performed to compare variables. All results were expressed as mean &plusmn; SD and P value &lt;0.05 was accepted as statistically significant.</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Brain Histological Study</div>\r\n<div>\r\n	Effect of 30 days lead exposure with TRF supplementation on the hippocampus</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A formal quantitative analysis by one-way ANOVA demonstrated that lead caused an increase in the number of apoptotic-like cells in the hippocampal subregion scornusammonis CA1 (F= 92.264, p&lt;0.05) and cornusammonis CA3 (F= 78.704, p&lt;0.05) (Table 1). Lead exposure was shown to cause histopathological changes in the hippocampus area cornusammonis CA1 (Figure 1) and area cornusammonis CA3 (Figure 2). Compared to the control (CTRL group), the group fed with 0.2% w/v lead acetate (PB2) showed neurons with apoptotic-like appearance with shrunken and condensed cytoplasm in addition to disorganized arrangement of neurons. TRF supplementation during lead exposure (PB2T group) was found to reduce the number of neuronal cells with apoptotic-like appearance at hippocampal subregion cornusammonis CA1 (Figure 1) and cornusammonis CA3 (Figure 2) when compared to PB2 group (P&lt;0.05). However, disorganized arrangement of neurons was still markedly observed in PB2T group ascompared to CTRL group.</div>\r\n<div>\r\n	Biochemical Study</div>\r\n<div>\r\n	Effect of 30 days lead exposure with TRF supplementation on brain lead level</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Brain lead levels were found significantly higher in group receiving 0.2% w/v lead acetate, PB2 group (14.20 &plusmn; 1.01 &mu;g/gm) compared to CTRL group (5.36 &plusmn; 1.27 &mu;g/gm) (p&lt;0.05). Brain lead level in TRF-supplemented group during lead exposure (PB2T group) was found significantly lower (4.07 &plusmn; 0.78 &mu;g/m) when compared to PB2 group (p&lt;0.05) (Figure 3).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Effects of 30 days lead exposure with TRF supplementation on brain TRF level</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Brain TRF levels were found significantly lower in group receiving 0.2% w/v lead acetate, PB2 group (0.24 &plusmn; 0.12 &mu;g/ml) compared to CTRL group (0.75 &plusmn; 0.26 &mu;g/ml) (p&lt;0.05). Brain TRF level in TRF-supplemented group during lead exposure (PB2T group) was found significantly higher (0.68 &plusmn; 0.19 &mu;g/ml) when compared to PB2 group (p&lt;0.05) (Figure 4).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Effect of 30 days lead exposure with TRF supplementation on erythrocyte SOD activity.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	No significant difference of erythrocyte SOD activity was found between groups receiving 0.2% w/v lead acetate, PB2 group (192.31 &plusmn; 25.01 U/mgHb) and CTRL group (177.19 &plusmn; 42.26 U/mgHb) (p&gt;0.05). Erythrocyte SOD activity in TRF-supplemented group during lead exposure (PB2T group) was found significantly higher (264.14 &plusmn; 36.32 U/mgHb) when compared to PB2group and CTRLgroup (p&lt;0.05) (Figure 5).</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Effect of 30 days lead exposure with TRF supplementation on brain MDA level.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Brain MDA levels were found significantly higher in groups receiving 0.2% w/v lead acetate, PB2 group (14.73 &plusmn; 2.96 &mu;M) compared to CTRL group (9.16 &plusmn; 1.35 &mu;M) (p&lt;0.05). No significant difference in brain MDA level was found between groups receiving TRF supplementation during lead exposure, PB2T group (9.84 &plusmn; 2.99 &mu;M) when compared to PB2 group (14.73 &plusmn; 2.96 &mu;M) (p&gt;0.05) (Figure 6).</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	The exact mechanism of lead (Pb) inducing toxicity has been of great interest in view of its major toxic effects on the central nervous system. An earlier study proved that TRF inhibits glutamate-induced activation of phospholipase A2 and causes neuroprotection. TRF is also believed as metal chealing agent (Khanna et al. 2010). A pilot study conducted prior to the present study concluded that thirty days lead ingestion even in a small dose of 0.2% w/v lead acetate through drinking water is capable of causing histopathological changes in the hippocampus of rats (Abd Aziz et al. 2012). The present study was conducted to determine possible protective effect of antioxidant tocotrienol-rich fraction (TRF) during lead exposure in order to better understand the mechanism involved in lead-induced brain toxicity. The present study provides evidence of decreased antioxidant enzyme activity and increased lipid peroxidation product as indices of oxidative stress in rats exposed to lead. TRF supplementation was shown to produce beneficial effects by increasing erythrocytes superoxide dismutase (SOD) activity and reduce lead accumulation in the brain of experimental rats. Histological observation in the present study provides supportive evidence of decresed histopathological changes in the brain tissue following TRF supplementation during lead exposure.</div>\r\n<div>\r\n	In relation to brain lead levels, results from the present study seemed to correlate with several studies reported to date. A similarly-designed study &nbsp;investigated the effect of green tea leaf extract (GTE) on animals treated with lead acetate 0.4% w/v for six weeks has reported significant decreased in brain and blood levels in the experimantal rats treated with antioxidant compared to non-treated group (Hamed et al. 2010). Additionally, the study showed positive correlation between brain lead levels and lipid peroxides (LPO) and negative correlations with glutathione (GSH). As also observed in the present study, reduction of at least two-folds difference of brain lead levels might be due to the capability of TRF to act as an antioxidant agents to excrete lead from the brain tissue. Earlier researchers given twelve weeks supplement of antioxidant N-acetylcysteine (NAC) to lead-exposed workers, reported significant reduction in blood lead levels in the NAC treated group. Oxidative stress status was found lower in the treated group compared to the non-treated group, due to the stimulated glutathione synthesis detected in the NAC-treated group (Kasperczyk et al. 2013). Several previous studies have shown that NAC, an orally available precursor of cysteine, is a chelator of toxic elements and may stimulate glutathione synthesis, particularly in the presence of vitamins C and E (Kannan &amp; Flora 2006; Blanusa et al. 2005).</div>\r\n<div>\r\n	In relation to the effect of TRF supplementation on the degree of lead-induced histopathological changes of brain tissue, the present study observed a significant decrease in number of cells with a typical morphology in TRF-treated group compared to non-treated group, although there were disorganized arrangement of cells in both groups. This might be due to TRF, even in the presence of lead at concentration used, possibly capable still to maintain the oxidant: antioxidant balance. This condition prevents excessive reactive oxygen species (ROS) production thus protect cells&rsquo; lipid membrane, protein and nucleic acid. Histological observation however suggests that TRF consumption at the concentration used is capable of reducing lead-induced apoptotic-like cells but not sufficient to maintain organized arrangement of cells. Earlier researchers have reported the protective effect of antioxidant ascorbic acid on the histopathological alterations in the 21-day old male pups of female rats administered with antioxidant ascorbic acid (100 mg/kg/day) during lead exposure (0.2% w/v) throughout pregnancy and lactation (Han et al. 2007). Increased risk of neuronal degeneration by lead-induced apoptosis was observed in immature and developing nervous system based on over expression of pro-apoptotic Bax gene, and antioxidant supplementation reported protective effects on pups&rsquo; hippocampal subregions cornusammonis CA1, cornusammonis CA3 and dentate gyrus in line with lowered levels of Bax protein and increased levels of Bcl-2 when compared to the non-treated group (Han et al. 2007). Additionally, a study on lead chloride (4 mM) interaction with selenium (Se) (60&mu;gm/kg body weight/day) reported marked reversing effect of both lead-induced alterations in neural cell adhesion molecules (NCAM), also concentration of lead in rat blood and hippocampus (Wang et al. 2013).</div>\r\n<div>\r\n	The present study showed significant high levels of TRF in the brain of TRF-supplemented group close to the levels of the control group, when compared to the non-treated group.TRF detected in the non-treated group therefore might have resulted from the amount of TRF that already exist in the rats&rsquo; body or obtained through diet given, however not sufficient to prevent the over production of free radicals which had lowered the brain TRF levels. Additionally, TRF levels in brain homogenate were also found to be negatively related to the brain lead levels. This is in accordance to a report by National Health and Nutrition Examination Survey III (NHANES III) on the possitive relationship between blood lead levels and oxidative stress markers in 10,098 adult subjects, in addition to negative correlations between blood lead levels and serum vitamin E, carotenoids and vitamin C (Lee et al. 2006). A study on the effect of vitamin E and vitamin C on lead exposure (500 mg/L) for 12 weeks to the liver and kidney of rats has reported a significant decrease of about thirty percents of initial TRF level, but not vitamin C when compared to the control group (Jurczuk et al. 2007). Beneficial effect of vitamin E (50 IU/kg body weight) intraperitoneally administered during lead exposure (12 mg/kg body weight) for 24 days has also been reported to prevent lead-induced testicular toxicity due to its antioxidant properties (Al-Masri 2015).</div>\r\n<div>\r\n	The present study evaluated the effect of TRF supplementation on the indices of oxidative stress in order to better understand the mechanism involved in lead toxicity. SOD enzyme activity per mg haemoglobin was determined in haemolysates of erythrocytes instead of brain homogenates. This is because SOD is present much greater in erythrocytes than in most tissues. Futuremore, previous literatures have shown that lead is capable in reducing SOD activity and other reactive oxygen species in erythrocytes (Mousa et al. 2002; Rendon-Ramirez et al. 2007). Erythrocyte SOD enzyme activitiy in TRF-treated group were found significantly higher compared to the control and non-treated group. This might be due to highly significant existing SOD enzymes in rats and in the presence of lead at the concentration used, able to neutralize excessive superoxide radicals produced by lead at the concentration used in the exposure. Besides, brain malondialdehyde (MDA) levels were also detected lower in the TRF-treated group compared to non-treated group. This might be due to TRF supplementation, at the concentration given, capable to protect cells&rsquo; lipid membrane from excessive superoxide radicals produced by lead, thus maintaining MDA levels similar to the control group. Effects of antioxidants like tocopherol, ascorbic acid and L-methionine following lead-induced toxicity to the liver, kidney and the brain of mice have shown that tocopherol (100 mg/kg body weight) significantly reduced lipid peroxidation products in both liver and brain tissues (Patra et al. 2001). A study on the effect of L-ascorbic acid and &alpha;-tocopherol on male rats exposed to lead has reported a decrease in nitric oxide and lipid peroxide levels to the normal level, in addition to a significant increase in SOD, glutathione peroxidase (GSHPx) and catalase (CAT) in both antioxidant-treated groups (Das &amp; Saha 2010). Indices of oxidative stress determined in the present study thus provide evidence of lead ability to produce oxidative stress in cells.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	The present study provided evidence of beneficial effect of tocotrienol-rich fraction (TRF) supplementation during lead exposure. TRF supplementation during lead exposure is capable of maintaining vitamin E at normal levels, reduces indices of oxidative stress, prevents histopathological alterations as well as reduces brain lead levels. Based on the limited parameters of antioxidant enzyme and product of lipid peroxidation determined in the present study, therefore further studies are needed to ascertain the exact action of lead in producing brain toxicity and other indices of oxidative stress following lead consumption.</div>\r\n<div>\r\n	Due to limited duration of the study and limited technique used, a few histomorphometric analyses along with quantitative analysis on apoptosis or necrosis of the brain cells were unable to confirm the event of apoptosis in the present study. Future studies should confirm the event of apoptosis using specific apoptotic assay.</div>\r\n<div>\r\n	Acknowledgements</div>\r\n<div>\r\n	The authors &nbsp;wish to thank the departments involved in the study, especially the Department of Anatomy, Faculty of Medicine, Universiti Kebangsaan Malaysia for providing the research grant UKM-GGPM-TKP-155-2010.</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(67688, 67674, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Polypoidal choroidal vasculopathy (PCV) is a retinal disorder characterized by the presence of out pouching or polyp like aneurismal dilatation of the choroidal vessels which is thought to be a clinical variant of neovascular age-related macular degeneration (AMD) (Yannuzzi et al. 1990; Ciardella et al. 2004; Gomi &amp; Tano 2008). PCV seems to preferentially affect pigmented individuals thus African-Americans and Asians are at higher risk. Various studies suggest that the incidence of PCV is considerably high in Asians, very high in blacks and low in whites. On the contrary, the incidence of AMD is high in whites; moderate in Asians and very low in blacks (Uyama et al. 2002; Sho et al. 2003). Most reports on the patterns of PCV in Asia are mainly based on patients from a homogenous race (e.g. Chinese, Japanese, etc) (Gomi &amp; Tano 2008; Uyama et al. 2002; Sho et al. 2003) and very few descriptions &nbsp;are from a mixed race population.&nbsp;</div>\r\n<div>\r\n	Malaysia, is a unique multiracial country, predominantly populated by the Malays (67.4%), followed by the Chinese (24.6%), and Indians (7.3%) where each race is unique in phenotypically and genetically (Department of Statistics Malaysia 2016). In the recent years, with increase awareness in macular diseases and in the current era of anti-VEGF treatment, Malaysia has seen a rise in the number of PCV patients.&nbsp;</div>\r\n<div>\r\n	The Universiti Kebangsaan Malaysia Medical Centre (UKMMC) is situated in the capital city Federal Territory of Kuala Lumpur; the most populous city in Malaysia. The centre is a tertiary referral centre, catering mainly for the population around the area, as well as receiving referral from various private and other local hospitals from all over Malaysia. Over the years it is noted that patients diagnosed with PCV in UKMMC appears to affect not only the Chinese population but other races too. This study therefore wanted to discover and describe the pattern of PCV in our mixed racial group particularly in terms of their features on fundus photographs, OCT, FFA and ICG. Then comparison made with other studies and see whether the findings differ or similar.</div>\r\n<div>\r\n	Materials and Methods</div>\r\n<div>\r\n	Patients were identified from the Department of Ophthalmology, UKMMC medical retina database. All patients with a confirmed diagnosis of PCV by two independent medical retina specialists from January 2013 to October 2015 were included in the study. Exclusion criteria were: exudative age related macular degeneration, high myopia, angioid streak, presumed ocular histoplasmosis syndrome, central serous chorioretinopathy, previous ocular surgery other than cataract and patient who were contraindicated for angiographic study. The medical records, fundus photo-graphs, optical coherence tomography, fundus fluorescein angiography and indocyanine green angiography of all patients were then retrospectively reviewed. Medical illnesses such as diabetes, hypertension, dyslipidemia and cardiovascular disease as well the smoking status were identified from medical records.</div>\r\n<div>\r\n	On fundus photography, the PCV manifestations were grouped into either serous or hemorrhagic PCV. In the serous PCV group, the findings included serous retinal detachment, serous PED and intraretinal lipid deposition. Hemorrhagic PED, subretinal and vitreous hemorrhage were grouped under hemorrhagic PCV. Presence of orange-like nodule as well as drusens were also documented.</div>\r\n<div>\r\n	Features of PCV on OCT that were observed were based on the description by Coscas (2009) and Sato et al. (2007). These included the the double-hump sign and a &lsquo;double-layer&rsquo; sign (Coscas 2009; Sato et al. 2007). Presence of exudative changes of the lesion and the loss of ELM and IS/OS interface were also looked at. &nbsp;</div>\r\n<div>\r\n	On FFA, features of &nbsp;expanding fluorescent leakage &nbsp;or pseudoclassic CNV lesion and location of this pseudoclassic CNV lesion were documented and classified either located at the subfoveal, juxtafoveal or extrafoveal region, adjacent or separate to the polypoidal lesion.</div>\r\n<div>\r\n	On ICG angiography, PCV was characterized by a branching vascular network (BVN) &nbsp;that lies anterior to choroidal vessels that can be seen on stereoscopic examination (Spaide et al. 1995). The location of the polypoidal lesions on ICG were assigned to either peripapillary (polyp and BVN located within 1500 um of the disc margin), macular (lesion is within 6000um diameter of the centre of FAZ) or extramacular (lesion lying outside the temporal vascular arcades) according to a previous study described by Lim et al. (2010) (Figure 1).</div>\r\n<div>\r\n	The polyp configurations were identified based on their angiographic features. The polyps were defined as solitary (one polyp) or multiple. If multiple, the polyps were classified as either cluster (or &lsquo;bunch of grapes&rsquo;), in a ring (or &lsquo;whorl&rsquo; pattern) or in a line (or &lsquo;string of pearls&rsquo;) (Lim et al. 2010; Cackett et al. 2009). In addition, we described two solitary polyps closely adjacent to each other as a diploid pattern (Figure 2). The study was approved by the UKM Research and Ethics Committee.</div>\r\n<div>\r\n	Statistical Analysis</div>\r\n<div>\r\n	Statistical analysis for descriptive statistics was performed using SPSS statistical software (SPSS, Inc, Chicago, IL, USA). &nbsp;Epidemiology and clinical and investigative features were described in percentages.</div>\r\n<div>\r\n	Results&nbsp;</div>\r\n<div>\r\n	A total of 91 eyes from 86 patients were included in the study. Patients&rsquo; age ranged from 54 to 86 yrs, (mean age of 70.4 yrs) for the whole study population. Almost two thirds of the patients were males (n=51, 59.3%) and 35 patients (40.7%) were females. Chinese race predominated (65.1%, n=56) followed by Malays (31.4%, n=27), Indians (2.3%, n=2) and Eurasian (1.2%, n=1). Thirty-five patients (40.7%) had diabetes mellitus as co-morbidities followed by 39 (45.3%) with hypertension, 31 (36.0%) had dyslipidemia and 12 (13.9%) had cardiovascular disease. Only 16 (18.2%) patients were active smokers. The presenting mean best corrected visual acuity (BCVA) was 0.78 &plusmn; 0.64 logMAR units (Snellen 6/36) and the majority of patients had unilateral presentation (94.2%, n=81).</div>\r\n<div>\r\n	Most of the clinical findings on fundus photography at the first visit were confined to the area of the macula (86.8%). The commonest retinal manifestations at presentation were subretinal hemorrhage, seen in 54 eyes (59.3%), followed by serous macular detachment in 49 eyes (54.4%) and pigment epithelial detachment in 49 eyes (53.8%) (Table 1). Forty eyes (43.9%) were found to have drusens in the affected eye (Figure 3) and 48 eyes (52.7%) had orange-like nodule.&nbsp;</div>\r\n<div>\r\n	On SD-OCT, 75.9% of the eyes had exudative changes and double-hump sign (n = 66 eyes), while 64 eyes (73.6%) had double layer sign on SD-OCT (Figure 4). Nearly all patients (97.7%, n=85) had loss of ELM and IS/OS interface. Other SD-OCT findings were summarized in Table 2.&nbsp;</div>\r\n<div>\r\n	FFA features of the CNV lesions were summarised in Table 3. On ICG, pulsatile polyps were seen in 15 eyes (18.8%) and the predominant location for these polypoidal vascular abnormalities was in the macula area seen in 79 eyes (86.8%). &nbsp;A cluster of grape-like (Figure 2) polypoidal vascular dilations were the most common polyp pattern observed in 46 eyes (58.2%) (Table 4).</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Apart from a previous report on visual outcome in vitrectomised PCV and AMD patients (Bastion et al. 2012), to the best of our knowledge this was the first study describing the pattern of PCV in a mixed race group of patients in a single tertiary centre in Malaysia. It was noted that the mean age in our study population was slightly older (70.4 years) compared to previous studies where mean age at presentations were within the age of 60. This was due to the fact that nearly a tenth (9.3%, n=8) of our PCV patients were 80 years old and above with the oldest being 86 years old. &nbsp;Although the majority of cases were among patients of Chinese-descendant (65%), one third of the cases in our study population were Malays (31%).</div>\r\n<div>\r\n	Our findings were consistent with several previous epidemiological studies where cases of PCV were reported to be more common in pigmented individuals such as blacks and Asians, (Yannuzzi et al. 1999; Ahuja et al. 2000; Yannuzzi et al, 1997; Lafaut et al. 2000; Kabasawa et al. 2011) and have a high incidence among the Japanese and Chinese populations (Table 5). In our multiracial study population, PCV occurrence had a preponderance for male gender (59.3%), unilateral involvement (94.1%) and macular location (86.8%) of the polyps and this was similar to other studies from Asia (Uyama et al. 2002; Yannuzzi et al. 1994). On the other hand, our findings differ greatly from the white population where PCV is reported to be more common among female patients with bilateral involvement and peripapillary location (Yannuzzi et al. 1990; Tsujikawa et al. 2007; Ojima et al. 2009; Lafaut et al. 2000). Important genetic substrate most probably explains the epidemiological similarities between our study population with other Asian populations and the difference observed with the white population. Further genetic studies however could be beneficial to explain and confirm this theory.</div>\r\n<div>\r\n	It is known that smoking is strongly associated with AMD and appears to be an important risk factor for PCV. In a study by Kabasawa et al. (2011), when compared to normal controls, smoking was associated with an odds ratio of 4.87 for PCV. Similar finding was demonstrated by Cackett et al. (2009) with an odds ratio of 4.4 for smoker. However in our patients, only 16 of them (18.6%) were active smokers and 10 patients (11.6%) were ex-smokers. &nbsp;</div>\r\n<div>\r\n	It had been previously reported that hypertension might be associated in one third of PCV patients (Spaide et al. 1995). Ahuja et al. (2000) and Lip et al. (2000) found in their studies that 23.5% and 20% of their patients had history of hypertension. Almost half of our patients (45.3%) had hypertension which may suggest a probable close relationship between hypertension and PCV.&nbsp;</div>\r\n<div>\r\n	In PCV, the clinical manifestations are similar to those of neovascular AMD (Table 1). Sub-retinal hemorrhage at the macula was the most common feature (59.3%) in our study group, followed by serous macular detachment and PED, 54.4% and 53.8% respectively. According to Yannuzzi et al. (1990), due to low level of subretinal fibrovascular proliferation and hyperplasia of the RPE in PCV, there is little subretinal fibrosis and disciform scarring in PCV. It was previously reported that subretinal fibrovascular disciform scarring is rare in PCV however; one third of our patients had subretinal fibrovascular proliferations in the macula. This can be due to late presentation of our patients prior to diagnosis. Subretinal fibrous proliferation causes severe irreversible visual loss due to the disruption of sensory retina and retinal pigment epithelium (RPE). In a study by Tsujikawa et al. (2007), PED was observed in 51 eyes (55%), of which 5 were hemorrhagic PED (9.8%) and 46 (90.2%) were serous. In our study, similar findings were observed where 49 eyes were found to have PED (53.8%), 13 eyes had hemorrhagic PED (26.5%) and 36 eyes (73.5%) had serous PED.&nbsp;</div>\r\n<div>\r\n	PCV is not usually associated with drusen, being reported only around 16.7% in a study by Yannuzzi et al. (1999) and around 26.7% in study by Scassellati-Sforzolini et al. (2001) among PCV patients. However, almost half (43.9%) of the PCV patients in our study population had drusen in the involved eye which is higher compared to other studies. In a recent study by Coscas et al. (2015), the authors postulated that there might be two distinct PCV groups which are: i) an idiopathic group (purely polypoidal) and ii) a subtype of neovascular (NV)-AMD group which is associated with drusen. They also found that the idiopathic group had a significantly better visual acuity between compared to the NV-AMD group. It is said that although these groups share some similarities; however the treatment approach and responses might differ based on the underlying pathogenesis (Coscas et al. 2015).</div>\r\n<div>\r\n	SD-OCT is an important investigative tool in the diagnosis of PCV as well in monitoring the disease progression and response to treatment. The specificity and sensitivity for the detection of PCV using SD-OCT is 92.9% and 94%, respectively (De Salvo et al. 2014). Actively evolving polyps are usually associated with exudative changes seen as sub-retinal or intra-retinal fluid collection. Bright hyper-reflective spots adjacent to the polyps can also be detected in the outer or inner retinal layers. The protrusion of the polyps can alter the outer retinal layers, with subsequent displacement of the outer nuclear layer. The external limiting membrane (ELM) and inner segment-outer segment (IS/OS) interface can be lost leading to severe visual loss (Coscas 2009). These OCT findings show that PCV lesions do not arise directly from the choroid (Balaratnasingam et al. 2016). These polypoidal lesions are mostly present at the margin and inside the PED, which may cause a &lsquo;notch sign&rsquo; between PED, also described as the &lsquo;double hump&rsquo; sign seen with OCT (Tan et al. 2015).&nbsp;</div>\r\n<div>\r\n	On FFA, almost 87% of eyes had polypoidal lesion adjacent to the CNV, as opposed to only 9% in the study by Sho et al. (2003). This suggests that in our patients, the pathogenesis of poly-poidal CNV in PCV might be similar to the CNV in AMD and not purely idiopathic in nature.</div>\r\n<div>\r\n	ICG angiography is very useful for diagnosing PCV as it often demonstrates polypoidal lesions with or without branching vascular network (BVNs) from the choroidal circulation. It also detects aneurismal and spheroidal vascular dilation of the choroidal vessel (Yannuzzi et al. 1990; Uyama et al. 2002; Yannuzzi et al. 1999). Majority of the polyps were located in the macula region in Asian patients (86.8% in the current study) as seen in our study, whereas it is equally distributed in the macular and peripapillary location in Europeans (Tsujikawa et al. 2007). In a study by Uyama et al. (2002), clustered grape-like polypoidal lesions were commonly observed in Asian population and it carries poor visual prognosis which is consistent with our study where more than half of our patients had clustered grape-like polyps on ICG and it would certainly affect the overall visual outcome of the patients in the study.&nbsp;</div>\r\n<div>\r\n	The present study was performed in a single tertiary centre. Thus, it may do not reflect the Malaysian population as a whole. In addition, we did not look at visual and treatment outcomes of these patients. It would also be interesting to compare the pattern of PCV presentation and investigations among races which we are currently conducting. &nbsp;Despite this, this study does provide a glimpse of the pattern of PCV among Malaysian.&nbsp;</div>\r\n<div>\r\n	In conclusion, the pattern in our mixed race community shows a difference in the pattern of PCV presentation. PCV in our population showed a tendency to be a subtype of neovascular AMD. This suggests that race may have a role in the manifestation of PCV and may influence treatment and PCV outcome in these patients.</div>\r\n<div>\r\n	Acknowledgement</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	The authors would like to acknowledge the technicians and photographers who performed the imaging of the fundus and took photos, angiograms and OCT. The authors also thank the nurses who helped out during the angiograms. The authors express their gratitude to Dr. Isa HM and Dr. Wong HS for their expertise in the image interpretations where needed.</div>\r\n', 1),
(67691, 67677, '<div>\r\n	INTRODUCTION</div>\r\n<div>\r\n	The scope of Complementary and Alternative Medicine (CAM) is extensive and heterogeneous. CAM encompasses all health systems, modalities, and practices other than those intrinsic to the politically dominant health system of a particular society or culture (Zollman &amp; Vickers 1999). The use of CAM for management of chronic disease is becoming increasingly popular (Furnham 2002; Baer 2007) and its usage can be influenced by health belief system, local culture and tradition, ethnicity, geography and family history. CAM practices may be unique to the locality, although some CAM products or practices are more known and recognized internationally, like acupuncture or the Ayurveda treatment. A survey in Australia showed that the prevalence of lifetime use of CAM was 85% (while the current consumption was 52%) and interest in CAM was evenly divided between urban and rural areas (Robinson et al. 2007). In Malaysia, Hasan et al. (2009) showed that the prevalence of CAM use among patients with chronic diseases in a hospital setting was also high at 63.9%.</div>\r\n<div>\r\n	Patients with chronic diseases are more likely to use CAM in disease management, compared to acute disease (van den Brink-Tuinen &amp; Rijken 2006). Since 1990s, studies on the prevalence of CAM use among patients with diabetes mellitus are growing and the data showed that CAM is a popular choice for diabetes management (Davis et al. 2011). Nonetheless, there are inconsistencies with formal definitions and categories for measuring the prevalence of CAM (Chang et al. 2007). Furthermore, the study designs varied widely, hence, the true picture regarding this issue is not as transparent as it could be. The available evidences suggest that there is a high prevalence of CAM use amongst diabetic patients and their use is complementary to the conventional medicine. However, patients are often less likely to disclose their CAM practice to doctors and health personnel (Willison et al. 2007). <span class="Apple-tab-span" style="white-space:pre"> </span>A study in Taiwan showed that the prevalence of CAM use increased tremendously among individuals, before and after being diagnosed with type II diabetes mellitus (Chang et al. 2007). The prevalence of CAM use was 22.7% before the diagnosis and soared to 61.0% following diagnosis. Chang and colleagues found that having diabetes is an independent predicting factor for the use of CAM. Similar finding was also found by another study (Villa-Caballero et al. 2010).</div>\r\n<div>\r\n	There is lack of local data on the use of CAM amongst diabetic patients in Malaysia. With the rapid rise of CAM usage amongst patients with chronic diseases, more studies are needed to identify the current pattern and trend of CAM use. The objectives of this study were to determine the prevalence of CAM usage and to identify the types of CAM commonly used by patients with type II diabetes in Negeri Sembilan.</div>\r\n<div>\r\n	MATERIALS AND METHODS</div>\r\n<div>\r\n	This was a descriptive cross-sectional study conducted in 45 government health clinics across Negeri Sembilan. Respondents at diabetes clinics were selected via systematic random sampling and interviewed using structured questionnaire. A placard with various examples of different categories of CAM was used to remind the respondents about CAM modalities that they might have used in the past 12 months. The inclusion criteria were patients with type II diabetes, aged 18 &nbsp;yrs and above, Malaysians and residents of Negeri Sembilan and registered with the National Diabetes Registry system. The exclusion criteria were patients with Type I diabetes and those who did not complete the questionnaire. Sample size calculation was based on two proportions cross sectional study formula with 10% non-response rate, making it a total of 856 respondents.</div>\r\n<div>\r\n	The questionnaire was developed based on literature review and two focus group discussions that were done before the survey. A pilot study was conducted with 30 participants at one health clinic. Face validation and content validation were conducted for the questionnaire.&nbsp;</div>\r\n<div>\r\n	CAM usage was divided into three groups; CAM use for diabetes (CAM-DM), CAM use for general health (CAM-G) and Non-CAM user. CAM-DM referred to respondents who used at least one CAM modality which he/she believed will help with his/her diabetes, for at least once a month for at least three consecutive months, in the past one year from the interview date. CAM-G referred to respondents who &nbsp;used CAM for general health or other medical illness but not for diabetes, for at least once a month, in the past 12 months from the interview date. Non-CAM user referred to respondents who did not use any CAM modality in the past 12 months from the interview date.</div>\r\n<div>\r\n	The data collected were analysed for descriptive analysis using Statistical Package for Social Sciences (SPSS) version 19.0.</div>\r\n<div>\r\n	This study was reviewed and registered with the National Medical Research Register (NMRR-12-884-12515) and also with the Universiti Kebangsaan Malaysia Medical Ethics and Research Committee (FF-330-2012).</div>\r\n<div>\r\n	RESULTS</div>\r\n<div>\r\n	A total of 828 respondents were interviewed from seven districts in Negeri Sembilan. The response rate from the sample size determined was 828/856, which was 97%. On the whole, the average age of respondents was 59 yrs. Nearly 60% of them were women and Malay ethnicity was the largest group at 46.5%. The majority of patients did not work but this is maybe due to the fact that the majority of them were housewives (Table 1).</div>\r\n<div>\r\n	The prevalence of CAM use among respondents in Negeri Sembilan was divided into three main groups (Table 2). In total, the prevalence of any CAM use (CAM-DM: 40.6% and CAM-G: 17.9%) amongst patients with type II diabetes mellitus was 58.5%. Therefore, almost two-thirds of patients with type II diabetes in Negeri Sembilan practiced CAM whether to help diabetes or for other purposes.&nbsp;</div>\r\n<div>\r\n	The main reason for the use of CAM products or practices in both CAM-DM and CAM-G groups was because that the respondents believed in the effectiveness of CAM (Table 3). Harmful side effects from prescribed conventional medications were the least chosen reason by respondents who used CAM.&nbsp;</div>\r\n<div>\r\n	Majority of patients in the CAM-DM group (28.9%) used 3 CAM products or practices for diabetes and/or for other health problems in the past 12 months (Figure 1). However, few patients also utilised up to 6-8 different modalities of CAM within a year. During the interviews, it appeared that they did not use them all at once, but rather like experimenting with different types of CAM modalities within a one year period. In contrast, with the CAM-G group, the majority 54.1% used one CAM product or practice only to help with general health or other ailments (Figure 2).&nbsp;</div>\r\n<div>\r\n	Within the CAM-DM group, bitter gourd (Momordicacharantia) was the most popular CAM used to help control diabetes (Table 4). Bitter gourd was used in many forms such as freshly blended, freshly cooked, dried, infused in hot water or bought as commercial products in the form of pills or drinks. In CAM-G group supplement milk was the most popular (Table 5). The most popular supplement milk brands were Anlene&reg; and Omega Plus&reg;.</div>\r\n<div>\r\n	When asked on whether they informed their healthcare provider about their CAM use, 78.9% of CAM-DM group and 65.5% of CAM-G group never informed the healthcare provider about their CAM use.</div>\r\n<div>\r\n	DISCUSSION</div>\r\n<div>\r\n	The use of CAM was popular amongst patients with type II diabetes mellitus in Negeri Sembilan. Patients may utilise CAM products or practices to help with diabetes management, for general health or for other ailments. Natural plant based products such as bitter gourds were the most commonly used CAM by those who wanted to control their diabetes.</div>\r\n<div>\r\n	There were more female respondents who used CAM and the proportion of male and female among CAM users was similar between the CAM-DM and CAM-G groups. However, among the Non-CAM users, distribution of gender was almost equal. There are several suggestions that might explain why there are more female CAM users. Parkman (2005) stipulated that women are more prone towards holistic approach towards health, including the use of CAM. Additionally, women were also more likely to take active role in personal health management (Wu et al. 2007).</div>\r\n<div>\r\n	The high prevalence of CAM usage found in this study is similar to other studies done in Malaysia. A cross-sectional study in an outpatient clinic in Ipoh found that 56% of patients with diabetes using CAM (Remli &amp; Chan 2003). Likewise, Ching et al. (2013a) found that the prevalence of CAM use among diabetic patients at a health clinic in Sepang was 62.5%. However, the prevalence in our study was slightly higher compared to the National Health and Morbidity Survey in Malaysia conducted in 2006 (NHMS III), in which the prevalence of CAM used in the past one year was 55.6% (Siti et al. 2009).</div>\r\n<div>\r\n	The high prevalence of CAM use amongst patients with type II diabetes in Negeri Sembilan was also consistent with the prevalence shown by other chronic disease patients in Malaysia. It was found that CAM use amongst patients with hypertension was 62.6% (Ching et al. 2013b). Amongst patients with asthma, the prevalence was 61.1% (Alshagga et al. 2013), while Hasan et al. (2009) showed that 63.9% of patients attending chronic diseases outpatient clinic used CAM in their health self-management.</div>\r\n<div>\r\n	CAM use is also evidently popular among diabetic patients in several countries in South East Asia. For example, the prevalence in Thailand is 47.8% (Moolasarn et al. 2005). In the Philippines, more rural patients using CAM (68.4%) compared with patients in the city (51.5%) (Dahilig &amp; Salenga 2012). The prevalence of CAM use is lower among diabetic patients in Singapore, with the prevalence of 43.4% (Fan et al. 2013).</div>\r\n<div>\r\n	Nearly a third of CAM-DM group used 3 types of CAM products while more than half of CAM-G group used only one CAM product or practice. In another survey done at a hospital in Malaysia, 64.4% of the CA users were found to be using only one type of CAM while 35.6% were using two types of CAM (Huri et al. 2009).</div>\r\n<div>\r\n	The most popular choice for the CAM-DM group was natural plant based products such as bitter gourd. A systematic review has demonstrated that the uses of natural products are often the primary choice of CAM practice (Birdee &amp; Yeh 2010). More than half of the respondents in the CAM-DM use bitter gourd or Momordica charantia to help control diabetes. A similar rate was shown by another study at a health clinic in Sepang (Ching et al. 2013a). In Sri Lanka, Medagama et al. (2014) found 62.8% of patients with diabetes mellitus used bitter gourd as supplements with the intention to help controlling the blood glucose level. A study on two local types of bitter gourd (peria katak and peria kambas) in Malaysia found that they both had hypoglycaemic effects through inhibition of &nbsp;&alpha;&alpha;-amylase and &alpha;&alpha;-glucosidase activities (Ee Shian et al. 2015).</div>\r\n<div>\r\n	There are many factors why natural CAM products are the main choice for many diabetic patients. Besides being easily available, cultural factor plays an important role, as well. The use of herbal and traditional medicine is already a norm for many patients in Malaysia. Siti et al. (2009) found that there was no significant difference across the three major ethnicity groups in the usage of biological-based therapies for health issues. According to Lee et al. (2004), a strong belief in traditional way of health managementis a predictive factor for CAM use amongst diabetics in Asia. In Malaysia, this was substantiated by Aziz and Tey (2009) who found that respondents with positive personal attributes and opinions towards natural remedies influence their likelihood of using herbal medicines such as bitter gourd.</div>\r\n<div>\r\n	A high proportion of CAM users believe in the effectiveness of CAM and this is because they feel that CAM use is more holistic for health (Robinson et al. 2007). Although, the medical efficacy of CAM in controlling diabetes is mostly unproven (Tackett &amp; Jones 2009), patients still believe that CAM could still contribute to the management of their diabetes. However, the majority of CAM users never informed the healthcare provider about their CAM use. The fear of healthcare providers rejecting their choice of health management might be the reason for informing about their CAM use. A study by Ismail &amp; Chan (2004) showed that few doctors discourage CAM as they believe that CAM is not beneficial for health management and it may causes harm towards the users. Healthcare providers should realise that many of their diabetic patients are complementing their clinical diabetes care with other supplementary products or practices and both parties should incorporate the discussion about CAM use when formalising patients&rsquo; diabetes care.</div>\r\n<div>\r\n	There are several limitations to this study. Many CAM products come with multiple health benefit claims and patients may use them to alleviate not just one illness. This study required respondents to recall CAM in the past one year and this may lead to recall bias. Having the interview done in clinic setting may also provoke Hawthorne effect on the respondents as they might feel CAM practice is not acceptable by the dominant health system.</div>\r\n<div>\r\n	CONCLUSION</div>\r\n<div>\r\n	The utilisation of CAM among patients with type II diabetes in Negeri Sembilan is common. Natural plant based products such as bitter gourds were the most commonly used CAM by those who wanted to control their diabetes. Majority of CAM users never informed the healthcare providers about their CAM use.</div>\r\n<div>\r\n	Acknowledgement</div>\r\n<div>\r\n	The authors would like to thank Dato&rsquo; Dr Zainal Ariffin Omar, ex-Head of Non-Communicable Diseases Section, Ministry of Health, Malaysia and Datuk Dr Zailan Dato&rsquo; Adnan, ex-Director of Negeri Sembilan State Health Department, for their contribution in assisting the conductance of this study.</div>\r\n', 1),
(67693, 67679, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Osteoporosis is defined as a systemic skeletal disease characterised by low bone mass and micro-architectural deterioration of bone tissue (CDC 1993). World Health Organization (WHO) defined osteoporosis based on the bone mineral density measurement by Dual energy X-ray Absorptiometry (DEXA) scan. Osteoporosis occurs when the bone mineral density falls more than 2.5 standard deviations below the standard reference for maximum bone mineral density of young adult female. Osteoporosis occurs more commonly in women, especially after menopause. Nevertheless, hip fractures due to osteoporosis has caused higher mortality rate in males than females (Amin &amp; Felson 2001). Furthermore, osteoporotic fracture in males was associated with significant morbidity and has huge impact emotionally and financially on their families and the society (Johnell et al. 2001).</div>\r\n<div>\r\n	There are three phases of fracture healing; the reactive phase, reparative phase and remodeling phase. In the reactive phase, platelets are activated to release products such as transforming growth factor-&beta; that triggers inflammation. At the same time, haematoma is formed to seal off the fracture area (Grundnes &amp; Reikeras 1993; Marsh &amp; Li 1999). In the reparative phase, granulation tissue is replaced by fibrocartilage and external callus is formed by intramembranous ossification at the periosteum. Finally, in the remodeling phase, the callus and vasculature are remodelled to their normal size and shape (Gerstenfeld et al. 2003). Osteoporotic fracture requires special attention as the healing may be impaired or delayed by the osteoporotic condition. In human, fracture healing is assessed clinically based on the range of motion and pain sensation. The fracture callous formed during healing may also be assessed using plain film X-ray. Clinical assessments may not be accurate due to variations in medical judgement, while plain X-ray is only a representative two-dimensional assessment (2D) of the three-dimensional (3D) callus structure.&nbsp;</div>\r\n<div>\r\n	DEXA is the gold standard imaging in making a diagnosis of osteoporosis (Hamdy et al. 2002; Winzenberg &amp; Jones 2011). DEXA scan works by passing a scanner which emits a dual energy beam of low dose X-rays over an area of skeleton and measuring it with a detector. Several studies had investigated the use of DEXA scan in assessing bone fracture healing. Eyres et al. (1993a) &amp; (1993b) reported that DEXA scan could be used to analyze bone fracture healing from 1 to 2 weeks after distraction osteogenesis. Reiter et al. (1997) and Maffulli et al. (1997) recommended the use bone mineral density (BMD) and bone mineral content (BMC) values derived from DEXA scans to monitor bone fracture healing of femur or tibia. Markel et al. (1990) assessed fracture healing after tibial osteotomies in dogs and found that DEXA had good correlations with torsional properties and calcium content of the osteotomies.&nbsp;</div>\r\n<div>\r\n	Peripheral quantitative or quantitative computed tomography (pQCT or QCT) is another imaging technique which could be used to assess fracture healing. It provides 3D measurements of the structure and mineralization of the fracture callus, and may provide reliable and non-invasive assessments of fracture healing in humans. QCT was reported to have strong correlations with local material properties, such as indentation stiffness and calcium content during fracture healing in dogs (Markel et al. 1990).</div>\r\n<div>\r\n	Lately, micro-CT has emerged as the new paradigm technique for measuring bone structure and strength in 3D. In small animal model, micro-CT is more suitable compared to pQCT and QCT as it provides superior resolution for studying bone fracture healing (Duvall et al. 2007; Gabet et al. 2004; Gardner et al. 2006; Geiger et al. 2005; Midura et al. 2005; Schmidhammer et al. 2006; Shefelbine et al. 2005). It provides an accurate insight into the bone mineral density changes and biomechanical properties for the estimation of bone strength and fracture risk (Boyd et al. 2006). Micro-CT imaging is useful in monitoring murine fracture calluses at multiple post-fracture time points and under different experimental conditions that alter fracture healing. It measures parameters such as bone volume, bone volume fraction, and mineral density in the fracture callus to predict the strength of callus formed during fracture healing. Therefore, micro-CT can be used for non-invasive assessments of fracture healing and for identifying biological and biomechanical mechanisms that lead to impaired or enhanced healing (Morgan et al. 2009).&nbsp;</div>\r\n<div>\r\n	In the present study, micro-CT was used to assess the osteoporotic fracture healing of orchidectomised rats which were administered synthetic testosterone and testosterone-like herb, known as Eurycoma longifolia Jack (EL). Testosterone is a steroid hormone that regulates bone remodelling and maintains bone mineral homeostasis. Testosterone deficiency or hypogonadism is the major cause of osteoporosis in men. Testosterone replacement is the main treatment for androgen-deficiency in men (Bhasin et al. 2010; Nieschlag 2006). It was reported that testosterone therapy improves bone mineral density in men (Nieschlag et al. 2004). However, long-term use of testosterone may increase the risk of prostate cancer (Ellem &amp; Risbridger 2006). Therefore, a suitable alternative for testosterone is required for the treatment of androgen deficiency in men and to promote osteoporotic fracture healing.</div>\r\n<div>\r\n	EL is a medicinal plant that is found along the slopes of hilly territories in the Malaysian rainforest (Burkill &amp; Hanif 1930). The water decoction of its root is used to enhance libido in men and as anti-aging supplements (Cyranoski 2005; Talbott et al. 2013). The herbal product is advertised in the internet and available on-line worldwide. EL has been discovered to contain eurycomanone, eurycomanol, eurycomalactone, and alkaloids that may help to increase the free testosterone level in blood and inhibit sex hormone binding globulin. The pro-androgenic effect of EL was demonstrated in male rat model (Ang &amp; Cheang 2001). In human, EL was reported to elevate testosterone level and increase semen volume (Tambi &amp; Imran 2010).&nbsp;</div>\r\n<div>\r\n	In the present study, the effect of testosterone replacement and EL on callous fracture in orchidectomised rat, the androgen-deficiency model was assessed using micro-CT.</div>\r\n<div>\r\n	Materials and methods</div>\r\n<div>\r\n	Forty-eight male Wistar rats weighing between 350 to 450 gms were obtained from the Laboratory Animal Resources Unit, Faculty of Medicine, Universiti Kebangsaan Malaysia (UKM). The rats were randomly assigned into sham-operated group (n=12) and orchidectomised group (n=36). Two weeks following orchidectomy or sham operation, the right tibiae of all the rats were fractured under anesthesia according to Stuermer et al. (2010). Briefly, an incision was made from the medial femur condyle to the middle of the tibia and osteotomy was performed at the metaphysis region using pulsed ultrasound (Piezosurgery&reg;, Mectron Medical Technology, Carasco, Italy). Following osteotomy, a T shaped titanium fixation plate XS (57-05140 Stryker Trauma, Selzach, Switzerland) was placed and fixed with 1.2 mm screws to stabilize the fracture.</div>\r\n<div>\r\n	The orchidectomised rats were further divided into three groups consisting of orchidectomised-control (ORX), orchidectomised and treated with EL extract (EL), and orchidectomised and treated with testosterone enanthate (TEN). The EL group was treated with daily oral gavages of 15 mg/kg of EL extract (Phytes Biotek. Sdn. Bhd., Shah Alam, Selangor). The TEN group received an intramuscular injection of testosterone enanthate (Jesalis Pharma, Germany) at the dose of 7 mg/rat once a week (Yarrow et al. 2008). The SHAM and ORX control groups received vehicles (oral gavages of normal saline and intramuscular injection of olive oil) only. Following six weeks of treatment, the rats were euthanized, the fixation plate was removed and the tibiae were extracted and stored in 10% formalin at room temperature (Figure 1).&nbsp;</div>\r\n<div>\r\n	The protocol was approved by Universiti Kebangsaan Malaysia (UKM) Animal Ethics Committee (UKMAEC: PP/FAR/2011/NORLIZA/30 NOVEMBER/414-NOVEMBER-2011-JUNE-2012).&nbsp;</div>\r\n<div>\r\n	Micro-CT assessment</div>\r\n<div>\r\n	The degree of fracture healing was measured using in vitro micro-CT (&mu;CT 80 scanner, Scanco Medical, Switzerland). The system is equipped with a computer and evaluation software discretion for image analysis. For analysis procedure, the tibiae were placed in a sample holder in a vertical direction with the epiphyseal head facing upward. The source of energy was set to 70 kVp and 114 &mu;A and high definition resolution was selected to acquire better and accurate image (Ramli et al. 2012). The filter used was 0.5 mm Al. The voxel size was 10 &micro;m and the number of slices included was 201.</div>\r\n<div>\r\n	The fracture callus area which consisted of 100 axial slices above and below fracture line was chosen as the region of interest (ROI) to be scanned (Figure 2). A set of 2D images in form of vertical slices was produced from the scanning process. From these 2D images, a 3D evaluation could be performed by contouring all the 201 cross-sectional slices resulting from the 2D data set within the ROI. The bridging cortices were then extracted by applying a threshold of 400 mgHA/cm3, Guassian noise filter of 0.8, and support of 2. This segmentation threshold was utilized in all analyses. Using the Scanco evaluation software, the parameters measured were fraction of mineralized tissue in bridging cortices (BVbc/TVbc), bone mineral density of the bridging cortices (mBMDbc) and soft callus volume (BVcallus).</div>\r\n<div>\r\n	Statistical Analysis &nbsp;</div>\r\n<div>\r\n	All the data were expressed as mean &plusmn; standard error mean (SEM). Statistical analysis was conducted using Statistical Package for Social Sciences version 18.0 (SPSS 18.0, Chicago, USA). Kolmogorov Smirnov test was used for normality testing. Normally-distributed data was analysed using parametric one-way ANOVA test followed by Tukey&rsquo;s HSD test while not normally-distributed data was analysed using Kruskall-Wallis, Mann-Whitney U and Wilcoxon Signed Rank tests. The level of significance was taken as p&lt; 0.05.</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	Three dimension (3D) images of bridging cortices of the callus during fracture healing</div>\r\n<div>\r\n	Bridging cortices refered to callus that bridge the fracture gap. When the 3D images of the bridging cortices of the calluses were compared, the ORX group showed the thinnest callus and poorly connected cortices compared to the other groups. The TEN group exhibited the thickest callus and marked connection between the cortices. The SHAM group displayed good connection of the cortices, but the callus thickness was lower than the TEN group. The EL group showed better cortices connection than the ORX group but more inferior compared to the TEN and SHAM groups (Figure 3).</div>\r\n<div>\r\n	Fraction of mineralized tissue of the bridging cortices (BVbc/TVbc)</div>\r\n<div>\r\n	This parameter measured the ratio of mineralised callus that bridged the fracture gap to the total callus. The BVbc/TVbc of the ORX group was significantly lower than the SHAM and TEN groups. The EL group seemed to have higher BVbc/TVbc than the ORX group but the difference was not statistically significant (Figure 4).</div>\r\n<div>\r\n	Mineralized Bone Mineral Density of Bridging Cortices (mBMD)bc&nbsp;</div>\r\n<div>\r\n	This parameter measured the bone mineral density of the callus that bridged the fracture gap. There were no significant differences in the mBMDbc among the groups (Figure 5).&nbsp;</div>\r\n<div>\r\n	Soft Callus Volume (BVcallus)</div>\r\n<div>\r\n	This parameter measured the volume of unmineralised callus. There was no significant difference in the soft callus volume parameter among the groups (Figure 6).&nbsp;</div>\r\n<div>\r\n	Discussion&nbsp;</div>\r\n<div>\r\n	Micro-CT has proven to be a useful tool for pre-clinical anatomic assessment of small animal models including cardiovascular system (Jorgensen et al. 1998; Langheinrich et al. 2004; Schambach et al. 2010; Badea et al. 2005), gastrointestinal system (Graham et al. 2008; Martiniova et al. 2010) and skeletal system (Dom et al. 2012; Jiang et al. 2000). Micro-CT can differentiate between bone and soft tissues and provide high resolution images for both cortical and cancellous bones (Jayusman 2015). With these capabilities, micro-CT is becoming the standard tool for bone evaluation including the healing of bone fracture (Liu et al. 2008).&nbsp;</div>\r\n<div>\r\n	Previous studies on orchidectomised rat model had demonstrated mixed findings on the ability of EL to prevent osteoporosis. Biochemically, EL supplementation was shown to prevent bone calcium loss in orchidectomised rats (Shuid et al. 2011). However, a histomorphometric study found that EL supplementation failed to protect bone against osteoporosis in the same model (Tajul Ariff et al. 2012). A micro-CT study by Ramli et al. (2012) found that EL supplementation at the dose of 90 mg/kg was able to preserve bone volume of orchidectomised rats but not at lower doses.&nbsp;</div>\r\n<div>\r\n	To date, no study has been carried out to determine the effects of EL on fracture healing of osteoporotic bone. In this study, fracture was induced in tibiae of orchidectomised rats to represent osteoporotic fracture of androgen-deficient osteoporosis model. Fracture was created at the metaphyseal region of the tibia as osteoporotic fracture occurs more frequently at this area in human (Thompson et al. 1995). Tibia was chosen to be fractured as there is only a thin subcutaneous tissue lining the anterior part of the bone with limited vessels. Therefore, trauma and haemorrhage, which could affect fracture healing, is minimal in the tibia fracture model. The osteoporotic fracture healing with testosterone replacement and EL was assessed using micro-CT.&nbsp;</div>\r\n<div>\r\n	Micro-CT imaging provides qualitative and quantitative analyses of bone such as bridging cortices fusion and volumetric BMD (Habermann et al. 2010; Nyman et al. 2009). &nbsp;As expected from the micro-CT analysis, the worst fracture healing was observed in the ORX group while the best healing was observed in the TEN group. Qualitatively, the EL group seemed to heal better than ORX group. However, the healing did not record any significant difference for the BVbc/TVbc, mBMDbc and BVcallus parameters measured. The best parameter to assess fracture healing is BVbc/TVbc as the number of cortices bridged by callus is the most objective measure (Whelan et al. 2002). The EL group has shown the best results in this important parameter compared to other two parameters which indicate some potential in promoting fracture healing.&nbsp;</div>\r\n<div>\r\n	Higher doses of EL may be required but based on a previous study; the dose used in this study should have been adequate (Shuid et al. 2011). &nbsp;Furthermore, the dose was nearly twice the dose of EL used to increase sperm counts in normal rats (Wahab et al. 2010).&nbsp;</div>\r\n<div>\r\n	The micro-CT parameters were sensitive enough to show reduced fracture healing in the ORX group and better healing with the TEN group. The effect of EL was not significantly captured by micro-CT. Orchidectomised rats may not be a suitable model to determine the effects of EL. One of the mechanisms to increase testosterone by EL is by elevating the luteinizing hormone (LH) and follicle stimulating hormone (FSH), which would stimulate the Leydig cells to produce testosterone in testes (Low et al. 2005). Since the rat&rsquo;s testes were removed in this model, the EL&rsquo;s testosterone-raising effect through this mechanism was not available.&nbsp;</div>\r\n<div>\r\n	In this study, micro-CT was proven to be the best imaging tool for non-invasive assessment of fracture healing. However, there were several limitations with micro-CT scanning. Frequent scanning especially with in-vivo micro-CT may lead to bone structural deformities. Lower radiation dose should be used but this may lead to reduce image sharpness and resolution. Therefore, there should be a balance between image quality and radiation risks. It was demonstrated that repeated micro-CT imaging for six weeks with a two week time interval at the radiation dose of 776 mGy and pixel size of 9 &mu;m had negative effects on the trabecular bone structure (Laperre et al. 2011).&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Based on the orchidectomised model, EL has shown potential in promoting fracture healing but was not as effective as testosterone. A micro-CT study on EL should be repeated with a different castration model.</div>\r\n<div>\r\n	<div>\r\n		Acknowledgements</div>\r\n	<div>\r\n		The authors would like to thank UKM and the Ministry of Higher Education for the grant (ERGS/1/2012/SKK03/UKM/02/1) and Encik Fadhlullah Zuhair Japar Sidek and other staff members of Pharmacology Department for their technical support.</div>\r\n	<div>\r\n		&nbsp;</div>\r\n</div>\r\n', 1);
INSERT INTO `content_field_fulltext` (`vid`, `nid`, `field_fulltext_value`, `field_fulltext_format`) VALUES
(67696, 67682, '<div>\r\n	Bone is a unique dynamic tissue whereby its components undergo continuous resorption and re-deposition by a process known as bone remodelling. Bone remodelling is a lifelong process which starts from the very earliest foetal bone formation and continues until death. In brief, this process involves the breakdown of mature bone (bone resorption) by osteoclasts and the activation of osteoblasts to build new bones (bone formation). Therefore, bone remodelling is a result of osteoclasts and osteoblasts activities (Abdul-Majeed et al. 2013). Bone resorption and formation are balanced in a homeostatic equilibrium to continuously replace old bones by new ones and to guarantee that there is no net loss or gain of bone; this coordinated balance is called coupling. The primary function of bone remodelling is to maintain bone health. However, uncoupling leads to unbalanced bone remodelling process and results in serious bone diseases like osteoporosis (Abdul-Majeed et al. 2015).</div>\r\n<div>\r\n	Bone histomorphometry is a quantitative method to obtain information on the process of bone remodelling at the level of the basic multicellular units. Static bone histomorphometry is used to examine bone histology and quantitatively evaluate the activity of bone cells at a specific time. Bone is a dynamic organ. Therefore, dynamic bone histomorphometry is the standard method for evaluating changes in cellular activity and bone remodelling over time (Vedi &amp; Compston 2003; Kulak &amp; Dempster 2010).</div>\r\n<div>\r\n	The Weibel technique is a quantitative stereological method for morphometric cytology and utilizes the M42 grid which is also known as a multipurpose test-system. Moreover, it can generate quantitative data on the internal structure of cell, tissue and organ (Weibel et al. 1966; Freere &amp; Weibel 1967).&nbsp;</div>\r\n<div>\r\n	Several preclinical studies had used the Weibel technique to determine static and dynamic bone histomorphometric parameters in intact rat and osteoporotic rats. Moreover, these studies used this technique to investigate the efficacy of antiosteoporotic therapies (Nazrun et al. 2005; Shuid et al. 2007; Hermizi et al. 2009; Mehat et al. 2010; Hermizi et al. 2011; Aktifanus et al. 2012; Fathilah et al. 2012; Hussan et al. 2012; Muhammad et al. 2012; Soelaiman et al. 2012; Abdul-Majeed et al. 2012; Chin et al. 2014; Parvaneh et al. 2015). These studies have referred to (Weibel et al. 1966; Freere &amp; Weibel 1967) in performing the protocol without a detailed description on how did they evaluate the parameters. Therefore, the aim of this paper is to provide a solid reference for those who will be doing the same analysis.</div>\r\n<div>\r\n	In this present paper, we describe the utilization of the Weibel technique to measure static and bone histomorphometric indices. Processing of decalcified and undecalcified bone sample is also discussed. In this brief technical report, the trabecular bone of rat femora was used to describe the technique. However, this technical report may be applied to other types of bone.</div>\r\n<div>\r\n	Processing of bone samples</div>\r\n<div>\r\n	Three adult Sprague Dawley rats were purchased from the Laboratory Animal Research Unit, Universiti Kebangsaan Malaysia. The rats were kept two per cage under 12 hrs light-dark cycles. The rats were fed commercial rat chow and tap water ad libitum. After one week of acclimatization, the rats were sacrificed and the left femur of each rat was dissected out and cleaned of soft tissue. It was fixed in 70 % alcohol for at least 72 hrs and the distal third was divided sagittally using bone cutting tools with a low-speed saw. The first half was decalcified and stained with haematoxylin and eosin for static bone histomorphometric analysis while the second half was processed for dynamic histomorphometric analysis (Figure 1-4).The study protocol was obtained from the UKM Animal Ethics Committee, (PP/FAR/2011/IMA/27-JANUARY/352-JANUARY-2011&ndash;DECEMBER-2012).</div>\r\n<div>\r\n	Principles and General Consideration of Weibel Technique</div>\r\n<div>\r\n	The stereological test system is composed of line system (21 lines) and point system (42 points) standards which are the basis for applying the principles of stereology. The line system represents the surface measurements while the point system reflects the volume measurement (Figure 5). The M42 grid was adjusted on the captured image and the parameters were calculated based on the Freere &amp; Weibel (1967) formulas. The computed values were obtained in ratio or percentage.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	i) The relative surface area (Sa) was calculated as follows:</div>\r\n<div>\r\n	Sa=2N/Lt</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	N=number of interactions between the test line and the surface of interest</div>\r\n<div>\r\n	Lt=total number of the lines (21)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ii) The relative volume (V) was calculated as follows:</div>\r\n<div>\r\n	V=P/Pt</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	P=number of interactions between the points and the surface of interest</div>\r\n<div>\r\n	Pt=total number of points (42)</div>\r\n<div>\r\n	Quantification of Bone Cellular Histomorphometry Using Weibel Technique</div>\r\n<div>\r\n	Bone static histomorphometric parameters that were measured using the Weibel technique are osteoblast surface/bone surface (ObS/BS %), osteoclast surface / bone surface (OcS/BS %), eroded surface/bone surface (ES/BS %), osteoid surface/bone surface (OS/BS %) and osteoid volume/bone volume (OV/BV %) (Figure 6-7; Table 1).&nbsp;</div>\r\n<div>\r\n	i) Osteoblast Surface / Bone Surface (ObS/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NLOb=number of interactions between test lines and osteoblast cells</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ii)Osteoclast Surface/Bone Surface (OcS/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; =</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; = &nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NLOc=number of interactions between test lines and osteoclast cells</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	iii) Eroded Surface / Bone Surface (ES/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;</div>\r\n<div>\r\n	&nbsp; =</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;=</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;</div>\r\n<div>\r\n	NLES=number of interactions between test lines and eroded surfaces</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	iv) Osteoid Surface/Bone Surface (OS/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; = &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; =</div>\r\n<div>\r\n	NLO=number of interactions between test lines and osteoids</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	v) Osteoid Volume/Bone Volume (OV/BV)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp; =</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;&nbsp;</div>\r\n<div>\r\n	&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NPO=number of interactions between points and osteoid</div>\r\n<div>\r\n	NPTb=number of interactions between points and trabecular bones</div>\r\n<div>\r\n	Quantification of Bone Cellular Histomorphometry Using Weibel Technique</div>\r\n<div>\r\n	In order to quantify the dynamic histomorphometric parameters the femurs need to be fluorescent-labelled with intraperitoneal injections of 20 mg/kg calcein at day 9 and day 2 before the rats were sacrificed.&nbsp;</div>\r\n<div>\r\n	Bone dynamic histomorphometric parameters that were used as example are single-labelled surface/bone surface (sLS/BS %), double-labelled surface/bone surface (dLS/BS %), mineralizing surface/bone surface (MS/BS %), mineral apposition rate (MAR &mu;m/day) and bone formation rate (BFR &mu;m3/&mu;m2/day) (Figure 8, Table 2). Only sLS/BS % and dLS/BS % will be calculated by using the M42 grid while the rest of the parameters will be derived (Figure 9).</div>\r\n<div>\r\n	Single-labelled surface is formed after the first calcein injection (at day 9 before sacrifice) while the double-labelled surface is formed after the second calcein injection (at day 2 before sacrifice). Double-labelled surface is darker than single-labelled surface. In general, sLS/BS parameter corresponds to poor bone formation while dLS/BS, MS/BS and MAR correspond to bone formation and mineralization.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	i) Single-labelled surface/bone surface (sLS/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NLsLS=number of interactions between test lines and single-labelled surfaces</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	ii) <span class="Apple-tab-span" style="white-space:pre"> </span>Double-labelled surface/Bone Surface (dLS/BS)&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	=</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	NLdLS=number of interactions between test lines and double-labeled surfaces</div>\r\n<div>\r\n	NLTb=number of interactions between test lines and trabecular bones</div>\r\n<div>\r\n	iii) Mineralizing Surface / Bone Surface (MS / BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	iv) Mineral Apposition Rate (MAR)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MAR=X/Y</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	X=the distance between the two labels</div>\r\n<div>\r\n	Y=days between the two calcein injections (7 days)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	v) Bone Formation Rate/Bone Surface (BFR/BS)</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	BFR/BS=(MS/BS)*MAR</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	MS/BS=mineralizing surface</div>\r\n<div>\r\n	MAR=mineral apposition rate</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	A vast amount of literature showed that the use of the Weibel technique is a very effective stereological test system and give reliable results on the analysis of bone static and dynamic histomorphometric parameters (Nazrun et al. 2005; Shuid et al. 2007; Hermizi et al. 2009; Mehat et al. 2010; Hermizi et al. 2011; Aktifanus et al. 2012; Fathilah et al. 2012; Hussan et al. 2012; Muhammad et al. 2012; Soelaiman et al. 2012; Abdul-Majeed et al. 2012; Chin et al. 2014; Parvaneh et al. 2015). However, a detailed description of the protocol had not been mentioned. According to our knowledge this is the first technical paper describes the implementation of Weibel technique in the assessment of bone histomorphometric indices.</div>\r\n<div>\r\n	However, there are several types of errors that might lead to false results such as processing of bone samples, the distribution of parameter of interest over the trabecular and the identification of each parameter of interest in the trabecular sections. Fixation of bone samples, thickness of trabecular sections and standardized staining protocol are the errors that affecting the quality of the of bone samples. Optimized technical skills could overcome the errors that might occur during the processing of bone samples (Weibel et al. 1966). Errors due to distribution of the parameter of interest over the trabecular bone can be overcome by randomizing the trabecular sections of each bone sample and quantifying at least three images for each trabecular section. Understanding the mechanism of bone metabolism can achieve good identification of parameter of interest. However, this method can easily be applied to bone histomorphometric measurements at the microscopic levels. This paper was not designed to compare this technique with other techniques.</div>\r\n<div>\r\n	A combination of bone histomorphometric measurements with the assessment of bone mineral density, micro computed tomography, bone radiography, serum levels of biochemical markers and histochemical measurements might give a better overall indication about bone health status and may be useful to predict the risk of fractures.</div>\r\n<div>\r\n	ACKNOWLEDGMENT&nbsp;</div>\r\n<div>\r\n	The authors would like to acknowledge Universiti Kebangsaan Malaysia for providing research grants FF-073-2011 and UKM-DLP-2011-047 to conduct the current work. The authors like to thank the Department of Pharmacology, Faculty of Medicine, Universiti Kebangsaan Malaysia for the technical support and laboratory facilities to conduct this study.</div>\r\n', 1),
(67701, 67687, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Solar retinopathy is an insult of the retina photoreceptors which results from direct or indirect exposure to sun light. The damage is postulated to be due to the exposure to the visible blue light and the ultraviolet radiation (Schubert 2013). It is commonly associated with sun gazing, solar eclipse viewing, religious rituals or prolonged exposure to ophthalmic microscope light (Yannuzzi et al. 1987).</div>\r\n<div>\r\n	There are several imaging modalities that can be used to detect the changes in solar retinopathy. Spectral domain optical coherence tomography (SD-OCT) remains an important imaging tool in showing the damage of the photoreceptor layers (Chen et al. 2012). Our following case utilized the high definition, spectral domain optical coherence tomography (SD-OCT) from Spectralis (Heidelberg Engineering, USA) to depict the exact layers of the macula pathology in solar retinopathy. Besides that, we also picked up changes from the infrared (IF) and the fundus auto fluorescence (FAF) which corresponded to each other.</div>\r\n<div>\r\n	Case report</div>\r\n<div>\r\n	A 55-year-old fruit picker presented with right gradual reduction in vision for the past 2 yrs. There were no other associated ocular symptoms such as pain, redness or swelling of the periorbital region. The patient denied any history of trauma or foreign body entry into the eye. His past medical and ocular histories were unremarkable. Due to his occupation however, he did admit that his job required him to constantly gaze up to the sky, while wrapping fruits in the farm where he had inevitably looked directly at the sun from time to time. He denied wearing any protective sunglasses at work.&nbsp;</div>\r\n<div>\r\n	Upon presentation, his visual acuity was 6/36 in his right eye and 6/9 in his left eye, with no relative afferent pupillary defect. Clinical examination revealed normal anterior segments bilaterally. On funduscopy however, the right fundus showed multiple deep hypopigmented lesions at the parafovea region with a mottled macula (Figure 1). The left fundus was normal.</div>\r\n<div>\r\n	Results</div>\r\n<div>\r\n	SD-OCT showed generalised outer retina thinning of the right macula with a fairly intact external limiting membrane (ELM). There was presence of a sudden disruption of inner and outer segments of photoreceptors at the fovea with cavitations. Segmentations of retinal pigment epithelium (RPE) interdigitations were noted. Otherwise, the RPE/Bruch&rsquo;s membrane complex was intact (Figure 2).</div>\r\n<div>\r\n	We proceeded by performing IR and FAF tests of the right eye. IR revealed multiple hypo-reflective areas at the macula region (Figure 3). FAF showed a rim of hypoautofluorescence surrounding the fovea with intervening area of hyperautofluorescence (Figure 4). FA was performed and did not show any obvious telangectatic vessels (Figure 5). ERG revealed flattened and reduced waves in both scotopic and photopic response. The OCT, IR, FAF, FA and ERG of the left eye appeared normal. In view of his exposure to sunlight and findings from the multimodal imaging, a diagnosis of right eye solar retinopathy was made.&nbsp;</div>\r\n<div>\r\n	This patient was followed up for duration of two years and his right vision had no improvement. Clinically, his fovea remained mottled and a repeat OCT showed similar findings. Due to his chronicity of sun exposure and the ocular findings, we counseled him that his right vision may not improve in future and advised him to use protective eyewear to protect his left eye.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Solar retinopathy is a disease of the outer retina layer. Although the disease commonly affects both eyes, there are previous reports of unilateral solar retinopathy similar to our patient (Sheth et al. 2013; Dhir et al. 1981). It is postulated that unilateral cases are attributed to the uneven reflex blinking of the eyes and one eye being the more dominant than the other, hence the dominant eye is affected more.&nbsp;</div>\r\n<div>\r\n	Several modalities can be used to detect the retina changes in this condition. Bechmann et al. (2000) first described the changes associated with solar retinopathy using time domain OCT where they found a hyper-reflective area affecting all layers at the fovea. Other changes that have been documented by various literature reviews include abnormal reflectivity at the outer foveal retina with or without retina thickening, disruption of the inner and outer segments of photoreceptors and hyporeflective area of the underlying retinal pigment epithelium (Khatib et al. 2014).</div>\r\n<div>\r\n	In our case, the patient&rsquo;s fundus showed a mottled macula which indicates the chronicity of the disease. OCT showed generalized thinning of the outer retinal layers with preservation of the inner retinal layers thickness. This is in keeping with the fundamental understanding that solar retinopathy manifests as an outer retinal pathology. Interestingly in our case, the ELM layer was preserved. Another interesting feature seen in the OCT of this case is the presence of cavitations at the photoreceptor layers, similar with the cavitation at the inner retinal layer seen in macular telangiectasia type 2 (Charbel Issa et al. 2013). We postulate that this is most likely due to the loss of the photoreceptor tissues.</div>\r\n<div>\r\n	Our patient also demonstrated a hyperreflective area on IR which corresponded with the rim of parafovea hypoautofluorence area seen on FAF. This finding was well depicted from the colour fundus photograph which revealed hypopigmented lesions. These changes however were not previously reported in other cases associated with solar retinopathy. Based on these adjunct multimodality images, we believe that solar retinopathy isnot only a disease of the outer retinal layers but could also involve the retinal pigment epithelium (RPE) and choroidal areas as evidenced by the changes in AF and in IR images respectively.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Solar retinopathy remains an occupational hazard to people who are chronically exposed to sun. Careful history and clinical examination are pivotal in obtaining the diagnosis. The use of multimodal imaging is useful in depicting the changes of the retinal layers and to further assist us in understanding the pathology better.</div>\r\n', 1),
(67704, 67690, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Penile amputation is a scarce condition reported as an isolated case (Gyan et al. 2010). &nbsp;The common cause is self-mutilating or as a result of an industrial accident. In the area of psychiatry, penile amputation may occur as a complication of self-mutilation due to a commanding auditory hallucination. We report a case of a total amputation of the penis in a patient with acute paranoid schizophrenia. He responded to the command of his psychopatology and acted by castrating his penis, and the penis was not be able to be constructed due to loss of the remaining part of the genital.&nbsp;</div>\r\n<div>\r\n	Case report</div>\r\n<div>\r\n	This case involved a 31-year-old divorce Rohingya refugee from Myanmar who was given a protection for 8 yrs under United Nations High Commissioner for Refugees (UNHCR) for staying in Malaysia. Patient was brought to Emergency Department in Universiti Kebangsaan Malaysia Medical Centre (UKMMC), Cheras, Kuala Lumpur for an alleged self-harm with total amputation of his penis. Examination under anesthesia was done and he was admitted inthe hospital for further treatment. The wound was cleaned and he was started on intravenous antibiotic Augmentin for 7 days. Later he was transferred to psychiatry wad for further observation. With regard to his past psychiatric history, the man was treated previously in Myanmar for history of psychotic disorder. He had history of hearing voices talking to him and commanding him to get rid of his penis because he belief that his genital was controlled by some external forces. He had past history of alcohol dependence for past few years but denied history of substance abuse and intake. He did not harbour any suicidal ideation, and he had no past history of self-harm behavior.&nbsp;</div>\r\n<div>\r\n	In the hospital, after he regained his consciousness, he was given tablet Sulpiride 200 mg ON, an antipsychotic agent to treat his hallucination and tablet Clonazepam 2 mg ON was started to help him for sleeping. In the ward, his psychotic symptoms subsided and no alcohol withdrawal noted.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	There are &nbsp;less scientific reports on the prevalence of genital self-mutilation. There were only few cases of genital self-mutilation which were reported in the English literature. No precise data are available on the prevalence of male genital self-mutilation, and such acts are presumably much more frequent than the small number of published cases would suggest.</div>\r\n<div>\r\n	The patient presented with history of alcohol dependence which resulted in the symptoms getting worse. Earlier researcher such as Nakaya (1996), stated alcohol dependence may contribute self-mutilation because of its tendency to remove inhibition. This case report concluded that although it is difficult to estimate the risk of penile or genital-castration in view of the heterogeneity of the respective patient groups with a different clinical diagnosis, the danger of such deeds should be kept in mind. This is when the above factors, especially a commanding auditory hallucination for self-castration are present in the patient.</div>\r\n<div>\r\n	Cases of genital self-mutilation seem to be a global phenomenon across racial groups, cultures and religions. Cases involving male far out number female (Schweitzer 1990). Major self-mutilations such as self-amputations are rare in psychiatric practice and most patients are psychotic during such acts. Common types of major self mutilation are damaging to the digits, the eyes or the genitals (Gojendra Singh et al. 2013). &nbsp;This factor manifested to this case was male, presenting with history of psychotic disorder and the action involved penile or genital self mutilation. Psychopathology presented was commanding auditory hallucination. This symptom was noted as one of the common leading underlying psychopathologies in majority of the reported cases (Ozan et al. 2010).</div>\r\n<div>\r\n	There was three known groups of genital mutilator which can be divided to group of patient who has psychotic disorder (e.g. schizophrenia), transversites group and a group of patient with complex religious or belief (Yadukul et al. 2015). Various risk factors have been identified in the few previous case reports of genital self-mutilation (Mishra &amp; Kar 2001). Those factors are guilt feeling associated with sexual conflict, disturbance of sexual identity, conflict over masturbatory activities or transsexual tendencies. Profound ambivalence towards adult sexuality, religious psychotic experiences, schizophrenia, affective psychoses, alcohol intoxication, personality disorders, or borderline personality disorder which are associated with genital self-mutilation.</div>\r\n<div>\r\n	conclusion</div>\r\n<div>\r\n	Psychotic disorder as discussed in the present report manifested with positive clinical symptoms which lead to penile amputation. This finding suggests that penile amputation may present in case of psychotic disorder specifically in this discussion as schizophrenia.</div>\r\n', 1),
(67708, 67694, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Tracheostomy is associated with numerous complications, some of which continue to be relevant after placement. The most frequent late complication is the development of granulation tissue that may cause airway occlusion or result in airway stenosis (Epstein 2005). Lack of awareness and knowledge of tracheostomy care among medical staff may cause harm to the patient and lead to serious life threatening complications. &nbsp;Here, we present a case of large hypertrophic skin of tracheostoma with tracheal granulation tissue causing malpositioning of tracheostomy tube (TT) and airway obstruction, secondary to prolong placement of a cuff TT without change of more than four yrs due to improper tracheostomy care.</div>\r\n<div>\r\n	Case Report</div>\r\n<div>\r\n	A 32-year-old male with poor socioeconomic status and family support sustained an open laryngeal injury following motor-vehicle accident in 2002 requiring tracheostomy. Since then, he was on non-cuff TT due to laryngeal stenosis and was under Otorhinolaryngology (ORL) team of hospital nearby follow-up. He was successfully decannulated about five yrs later.&nbsp;</div>\r\n<div>\r\n	In 2011, about four yrs after decannulation, he presented again to another hospital with dyspnoea and reduced effort tolerance. A revision of tracheostomy was performed and subsequently he was sent back home with a non-cuff TT and given follow-up under ORL team.&nbsp;</div>\r\n<div>\r\n	Unfortunately, in 2012, he was involved in another motor-vehicle accident where he sustained injury to the left hand requiring a surgical procedure under general anaesthesia. During the procedure, his TT was changed to a cuff type for the purpose of mechanical ventilation. A few days after the procedure, he was sent home with the cuff TT size 6 without prior consultation with ORL team.</div>\r\n<div>\r\n	Since then, he had been using the same cuff TT without ever change it for about four yrs until recently he developed dyspnoea with noisy breathing.</div>\r\n<div>\r\n	Physical examination revealed large hypertrophic skin over peristomal area on the anterior neck causing the TT protruded anteriorly (Figure 1a &amp; 1b). Flexible nasopharyngolaryngoscopy showed distorted supraglottis and glottic stenosis with vocal cords at different vertical level (Figure 2). Flexible scope via tracheostomy tube at the stoma revealed presence of granulation tissue at the distal tip of TT obstructing 80% of the airway. The TT also adhered to the peristomal hypertrophic skin and was unable to be removed.</div>\r\n<div>\r\n	He was subsequently referred to our centre for further management. He underwent direct laryngoscopy and excision of peristomal hypertrophic scar. At the beginning of the procedure, TT was successfully mobilized, removed and changed to microlaryngeal tube for ventilation, following peristomal infiltration with marcaine adrenaline. The TT looked very dirty and mouldy (Figure 1c). The hypertrophic skin was excised by employing an elliptical incision around the lesion. Direct laryngoscopy revealed massive suprastomal granulation. At the end of the procedure, a new cuff TT tube was inserted that managed to bypass the distal granulation tissue.</div>\r\n<div>\r\n	He was then referred to the hospital social welfare team to assist him in term of financial support, hospital bill settlement and payment for new double lumen non-cuff TT. &nbsp;After few days of observation, TT was changed to new double lumen non-cuff type size 7. Subsequently, he was discharged home well from ward with the new TT and a date of follow-up was set for him.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	A number of mechanisms may cause late complications after tracheostomy (Stauffer et al. 1981). Granulation tissue is a common consequence of tracheostomy, occurring in 10-80% of cases, and it typically appears as pink and fleshy protuberant tissue produced by inflammation (Yaremchuk 2003). The common sites of occurrence are at suprastomal, infrastomal in the area of the cuff and the distal tip of TT.&nbsp;</div>\r\n<div>\r\n	Risk factors associated with granulation tissue formation include improper cannula size fitting, mechanical irritation of surrounding tissue caused by excessive tube movement, &nbsp;infection occurred at surrounding tracheostoma area, prolonged placement of TT, and anterior tracheal cartilage excision incommensurate with skin incision during the creation of the tracheostoma (Sue &amp; Susanto 2003). &nbsp;</div>\r\n<div>\r\n	The site where the TT balloon cuff placed within the tracheal lumen is the common site of granulation tissue formation. When the cuff pressure exceeds the tracheal wall capillaries perfusion pressure, it may induce ischemic injury. Prolonged ischemia may subsequently cause mucosal ulceration, chondritis, and cartilaginous necrosis, leading to the formation of granulation tissue. The insults to the mucosa may be further aggravated by shearing forces from the tube or cuff of the TT (Epstein 2005). Other areas in which granulation tissue prone to occur is near the distal tip of the TT that commonly caused by malpositioning of the TT. The tube malposition may increase the contact point between the tip and surrounding tracheal wall mucosa, predisposing it to granulation tissue formation. In obese patients with thick soft tissue in the anterior neck, the posterior tracheal wall mucosa is commonly affected.</div>\r\n<div>\r\n	In the present case, the patient was not obese but the prolonged placement, formation of anterior neck hypertrophic skin and a single use of cuff TT without changing for more than four yrs in the patient, predisposed him to tracheal mucosal irritation and infection which lead to mucosal ulceration, inflammation and granulation tissue formation. In normal practice, the tube has to be changed to a non-cuff preferably a double lumen before allowing the patient home.&nbsp;</div>\r\n<div>\r\n	Prevention of stomal infection is important as it may promote granulation tissue (Sasaki et al. 1979). Fewer complications due to granulation tissue were reported when TTs were changed twice weekly (Yaremchuk 2003). However, based on a Swedish study of long-term material wear of 3 different types of TTs made of silicone (Bivona), polyvinyl chloride (Shiley), and polyurethane (Tracoe), routine TT change every three months is recommended (Backman et al. 2009). They found biofilm formation on the TT, which can affect the structural integrity of the tube and cause damage. Substantial surface degradation was observed on TTs kept in place for three or six months, but no significant differences were established after three or six months of exposure between the various materials (Backman et al. 2009).</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	We learnt that suboptimal management for a patient on TT may lead to life threatening airway complications due to formation of peristomal hypertrophic skin causing malpositioning of TT and tracheal granulation tissue. Therefore, it is imperative to highlight this case to create awareness among medical personnel the importance of having basic knowledge on tracheostomy care. We recommend that the trained healthcare professionals in particular the ORL team must be engaged in managing patients with TT to ensure the long-term care.</div>\r\n', 1),
(67715, 67701, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Neonatal nasal obstruction is a rare cause of Congenital Nasal Pyriform Aperture Stenosis (CNPAS) in newborn as compared to posterior nasal obstruction, seen in choanal stenosis or atresia. Due to the narrowing at the anterior bony portion of nasal cavity (Rozner 1964), an infant usually presents with respiratory distress, cyclic cyanosis, episodic apnoeas, and feeding difficulties.</div>\r\n<div>\r\n	Confirmation of the diagnosis can be made by using Computed Tomography (CT). In order to assess the level of obstruction, thin axial section scans through the midfacial region is required. CT must be taken from the level of palate to the orbital roof. In term baby, a pyriform aperture width of less than 8mm is considered diagnostic of CNPAS (Belden et al. 1999). CT is also useful to differentiate between CNPAS and choanal atresia as both may present with similar signs and symptoms.</div>\r\n<div>\r\n	The pathogenesis of CNPAS is unknown. Various theories existed regarding its relationship and degree of association with craniofacial anomalies. There are isolated CNPAS cases reported as well as cases with association to chromosomal abnormalities. Hypothalamo-Pituitary Axis abnormalities and Solitary Median Maxillary Central Incisor (SMMCI) are very common (40% and 55%, respectively), followed by cardiac, vertebral and other brain anomalies (17.5, 12.5 and 30%) (Guilmin-Crepon et al. 2006).&nbsp;</div>\r\n<div>\r\n	Here, we report the first case of CNPAS in a baby with Trisomy 8 Mosaicsm chromosomal abnormality.</div>\r\n<div>\r\n	Case report</div>\r\n<div>\r\n	A full term baby boy, with birth weight of 2.23 kg, length 49 cm and head circumference 29.5 cm was born through emergency caeserean section for failed induction of labour. He was the second child in the family. Antenatally, his mother had gestational diabetes mellitus on diet control. The neonate was cyanosed with poor breathing effort and the appearance, pulse, grimace, activity, respiration (APGAR) score of 6 at 1 min, followed by 8 at 5 mins. The child was dependent on oxygen and was put on headbox oxygen 5L/min after birth till day 10 of life and was tapered down to nasal prong oxygen 2L/min. He was referred to the Otorhinolaryngology Department at 10 hrs of life for nasal obstruction.</div>\r\n<div>\r\n	Clinically, the infant had dysmorphic features such as small chin, widened forehead, low set ears and left hand polysyndectily (Figure 1). There was no cleft lip or palate. The nasal vestibular openings were narrowed and there was marked decrease in mist formation on spatula test bilaterally suggesting obstruction. Nasogastric tube size 6F and 2.7 mm paediatric flexible nasopharyngolaryngoscopy were introduced bilaterally but both were unable to pass through the nasal cavity.&nbsp;</div>\r\n<div>\r\n	Other congenital abnormalities seen in this infant were coarctation of aorta with large patent ductus arteriosus, mild to moderate hypoplasia of ventricles, bilateral undescended testes and bilateral multicystic dysplastic kidneys. &nbsp;Chromosomal studies confirmed Trisomy 8 Mosaicism. The analysis revealed 1 chromosome 8 showed an additional of unknown origin at 8p23.3. This abnormality imports monosomy for the region 8p23.3-&gt;8pter and trisomy for the additional chromosome material.</div>\r\n<div>\r\n	The CT paranasal sinuses showed inward bowing of bilateral maxillary spines and bony overgrowth of maxillary nasal process causing narrowing of the pyriform aperture with soft tissue obscuring the pyriform aperture. Pyriform aperture measured 5.4 mm at the level of inferior meatus (Figure 2). Vomer was normal and the distance of lateral wall of nasal cavity to vomer was symmetrical at 0.5 cm bilaterally.&nbsp;</div>\r\n<div>\r\n	The infant was treated conservatively. Nasal patency was improved with the use of saline solutions. He improved symptomatically and was discharged at 2 months of life. Clinically child was well with no cyanosis or respiratory distress. A nasogastric tube size 6 was able to pass through bilateral nares upon review.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Trisomy 8 mosaicism (T8M) also known as Warkany syndrome is an extremely rare entity, which involves chromosomal abnormality where there is an extra copy of chromosome 8. The incidence was estimated to be about 1&thinsp;:&thinsp;25000 to 1&thinsp;:&thinsp;50000 births (Wisniewska et al. 2002). &nbsp;It can occur with or without mosaicism. T8M is a human chromosomal disorder caused by three copies (trisomy) of chromosome 8. Complete trisomy 8 is usually associated with severe defect on the developing foetus and it causes 0.8% cases of miscarriages. However, individuals with T8M has variable phenotype ranges and would likely to survive into childhood and adulthood. The phenotype includes abnormal facies, eye anomalies, camptodactyly, reduced joint mobility, deep plantar, palmar creases, various vertebral and costal anomalies (Agrawal &amp; Agrawal 2011). &nbsp;</div>\r\n<div>\r\n	To date, and &nbsp;to the best of our knowledge, CNPAS has not been described in Trisomy 8 Mosaicsm. A midline developmental defect like holoprosencephaly which includes facial cleft is the most common cited association. However, in this case, the infant had no cleft lip or cleft palate. &nbsp;The least severe form of holoprosencephaly is single central maxillary incisor (SCMI). Arlis and Ward reported four out of six patients with CNPAS presented with SCMI (Arlis &amp; Ward 1992). Neither holoprosencephaly nor single central maxillary incisor is a common feature of Trisomy 8 Mosaicsm.&nbsp;</div>\r\n<div>\r\n	Neonatal nasal obstruction is a potentially life threatening condition as neonates are obligate nasal breathers. It may present with severe birth asphyxia. The differential diagnosis includes traumatic lesions, cysts, meningoencephalocele and encephalocele, tumours and choanal atresia. The latter can be a part of CHARGE syndrome (coloboma of the eye, heart defect, atresia of the nasal choanae, retardation of growth, genital abnormalities, ear abnormalities and deafness) (Al Abri et al. 2008).</div>\r\n<div>\r\n	CNPAS is a rare cause of nasal obstruction. The etiology and its pathogenesis remains unknown. Nasal pyriform aperture is made up of nasal and maxillary bones. Overgrowth of maxillary nasal process during maxillary ossification causes narrowing of the nasal aperture (Al Abri et al. 2008). Axial CT scan of the nose and paranasal sinus can assess the extent of the obstruction. Confirmation of diagnosis is made when the width of the whole pyriform aperture is less than 8mm and each pyriform aperture is less than 3mm (Arlis &amp; Ward 1992). Treatment for nasal pyriform aperture in general is either medical or surgical, depending on the severity of the symptoms. Eighty percent of the children with this problem (n=12) were treated surgically in the first year of life (Losken et al. 2002). In this case, the infant was treated conservatively and upon review at two months of age, he had no respiratory distress and was tolerating orally well.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	This was the case of CNPAS in a baby with Trisomy 8 Mosaicsm chromosomal abnormality. The clinical features and treatment disscussed in this case may be beneficial in future clinical practice.</div>\r\n', 1),
(67717, 67703, '<div>\r\n	Introduction</div>\r\n<div>\r\n	In the absence of direct trauma, certain fractures are suggested to be pathognomonic of seizures or electrocution, such as bilateral posterior fracture dislocation of the shoulders (Brackstone et al. 2001). Forceful muscular contractions as a result of convulsive seizures contributing to various types of fractures and dislocations have been reported; which includes the vertebras, hips, shoulders and jaw (Brackstone et al. 2001; Cagırmaz et al. 2015; Gosens et al. 2000; Lasanianos &amp; Mouzopoulos 2008; O&rsquo;connor-Read et al. 2007). Unilateral fractures may be caused by direct mechanical trauma, but convulsions occurring in epilepsy or electrocution are the most common cause of simultaneous bilateral fractures (Kristiansen &amp; Christensen 1984).</div>\r\n<div>\r\n	Generally, patients presenting to the Emergency Department post seizures without direct trauma, rarely present with complications of fractures. However, an orthopaedic complication should not be undermined and patients should be evaluated for fractures or dislocations after a seizure episode.</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Case Report</div>\r\n<div>\r\n	A 58-year-old lady who has been treated for epilepsy for the past 10 years was at home when she had 2 episodes of seizures, both 2 hrs apart. According to her children who witnessed the seizures, the patient was sleeping on the bed when the seizures occurred. &nbsp;The patient had generalized tonic-clonic seizures, which lasted for 5 mins each, associated with up rolling of eyeballs and drooling of saliva. She was in a post-ictal state for an hour, following the seizures. Several hrs later, the children decided to bring the patient to the hospital as she was unable to get out of bed and complained of generalized body aches.</div>\r\n<div>\r\n	On arrival at the Emergency Department, she was alert, but refused to move or talk much. On further history, she had one week history of fever with upper respiratory tract infection. She was independent and compliant to her anti-epileptic medication which was tablet Epilim 200mg twice daily. Eyewitness claimed there was no direct trauma during the seizure episode.</div>\r\n<div>\r\n	On physical examination, her vital signs were: blood pressure, 153/75 mmHg; pulse, 90 beats/min; respirations, 16/min; temperature, 37.1˚C. Blood glucose level was 8.3 mmol/L. General assessment revealed an alert and conscious lady who was grimacing in pain. She was unable to pinpoint the exact location of the pain, but claimed she was unable to move both her upper and lower limbs. Initial examination of her heart, lungs and abdomen were unremarkable.</div>\r\n<div>\r\n	The patient was prescribed some analgesia to relieve her pain, which was initially thought of as body aches secondary to the seizures. After some time, the patient was able to move her lower extremities, but still complained that her shoulders were stiff and painful.</div>\r\n<div>\r\n	More detailed examination of the extremities revealed that there was fullness in both the shoulders, which were symmetrical. No obvious deformity was seen and there were no lacerations or bruising. On palpation of the shoulders, humeral heads were within the glenoid fossae and there were no bony crepitus felt. The patient was unable to perform abduction, adduction, flexion, extension or rotation in either shoulder. Neurovascular status of the upper extremities was intact bilaterally. Examination of her lower extremities revealed no abnormalities and she was able to actively move them without any pain or restrictions.</div>\r\n<div>\r\n	Anteroposterior (AP) radiographs of each shoulder were ordered and revealed bilateral head and anatomical neck of humerus fractures (Figure 1 &amp; 2). The rest of her blood works revealed no electrolyte imbalances, while infective markers were not raised. She was subsequently referred to the orthopaedic team for further intervention. Bilateral U-slabs were applied. Open reduction and internal fixation with Philos plate were done for both humerus (Figure 3 &amp; 4). Intra-operatively bilateral comminuted fracture of proximal humerus was found. She was later discharged with no post-operative complications.</div>\r\n<div>\r\n	The patient was followed-up a few months later at the orthopaedic clinic for her fractures and the neuromedical clinic for her epileptic disease. She was seizure free and compliant to her anti-epiletic drugs. A year after the operation, the range of movement of her shoulders is still limited but she has no issues with activities of daily living.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	Bilateral proximal humerus fracture with shoulder dislocation is rare in seizure. Roux et al. in 2012 reported that three percent of neck of humerus fracture are associated with epilepsy (Roux et al. 2012). It is also known that posterior shoulder dislocation is more common following an epileptic seizure (Court-Brown et al. 2001). Typically, squaring of shoulder in a post ictal patient suggests presence of fracture with associated shoulder dislocation. However, this was not present in our patient which made the diagnosis of a fracture dislocation rather challenging. Persistent pain despite analgesia raised our suspicion which was later confirmed as bilateral head of humerus fracture radiologically. Seizure related proximal humeral fractures are almost always associated with dislocation of the glenohumeral joint, anterior and posteriorly (Noachtar 1998).</div>\r\n<div>\r\n	During convulsions, the shoulder joints are typically adducted, internally rotated and flexed, while scapular region muscles pull on the humeral head (Kristiansen &amp; Christensen 1984). When the shoulder girdle muscles (infraspinatus, teres minor, deltoid, latissiumus dorsi and teres major) contract with unbalanced force, it causes the humeral head to move superiorly and posteriorly. Humeral head fracture occurs due to the continuous pressure against the glenoid rim during seizure activity. Thus, these two combined mechanisms will lead to shoulder fracture dislocation (Brackstone et al. 2001; Gill et al. 2015).</div>\r\n<div>\r\n	Chronic use of antiepileptic drugs can increase catabolism of vitamin D and reduce calcium absorption leading to increase serum parathyroid hormone levels, and increase bone turnover. It is reported that the use of antiepileptic drug can lead to the development of pathological fractures by reducing bone mineral density (Wirrell 2006). Increased load on the osteoporotic bones during forceful muscle contractions in seizures can increase the risk of fractures. This may be true in our case, as the patient&rsquo;s long-term usage of an antiepileptic drug may have caused the decrease in bone mineral density, which is associated with a risk of fracture.</div>\r\n<div>\r\n	Emergency department doctors should have high index of suspicion towards fractures or dislocations of the extremities, even without significant history or physical evidence supporting it. A complete evaluation should be performed when the patient has recovered from the post-ictal period (Friedberg &amp; Buras 2005). Early recognition and treatment hence will improve prognosis and outcome.</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	Seizures in patients with epilepsy can cause non-direct traumatic fractures and dislocations of the extremities and the diagnosis of it is of importance for early referral and fixation to avoid ongoing patient discomfort and long-term morbidity. This case report was to highlight the uncommon occurrence of bilateral proximal humerus fracture after an epileptic seizure and the importance of secondary evaluation of post seizure patients presenting to the Emergency Department, in order not to misdiagnose fractures or dislocations of the extremities.&nbsp;</div>\r\n<div>\r\n	Conflict of Interest</div>\r\n<div>\r\n	The authors declare that they have no competing interests.</div>\r\n', 1);
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(67723, 67709, '<div>\r\n	Introduction</div>\r\n<div>\r\n	Cytomegalovirus (CMV) retinitis is an opportunistic eye infection characterized by dense, white confluent opacification of the retina along the vessels with retinal haemorrhage and vascular sheathing. It predominantly occurs in patients with acquired immunodeficiency syndrome (AIDS) with CD4 cell counts less than 50 cells/mm3. It has also been reported in Human Immunodeficiency Virus (HIV)-seronegative patients with systemic autoimmune disorder requiring systemic immunosuppression, organ or bone marrow transplantation; haematological or breast malignancy receiving chemotherapy; ocular diseases following intraocular or periocular corticosteroid injection; diabetes mellitus and Good syndrome (Kim et al. 2007; Han et al. 2015; Downes et al. 2016).&nbsp;</div>\r\n<div>\r\n	Dermatomyositis is an idiopathic inflammatory myopathy with characteristic skin manifestations. It has a strong association with malignancy including ovarian, lung, breast, pancreatic, stomach, colorectal, nasopharyngeal cancers, and non-Hodgkin lymphoma (Olazagasti et al. 2015). The reported incidence of cancer associated with dermatomyositis varies from less than 7% to over 30% (Di Rollo et al. 2014).</div>\r\n<div>\r\n	CMV retinitis in patients with i) concurrent dermatomyositis and malignancy, ii) cancer other than haematological or breast malignancy, or iii) cancer prior to chemotherapy has not been previously reported. We describe a case of CMV retinitis in a HIV-seronegative patient diagnosed with dermatomyositis and lung malignancy.&nbsp;</div>\r\n<div>\r\n	Case report</div>\r\n<div>\r\n	A 40-year-old woman with dermatomyositis complained of right eye blurred vision and floaters for three weeks. She was on oral Azathioprine 100mg OD, oral prednisolone 15mg OD and hydroxychloroquine 300mg OD for 13 months. Examination revealed right vision of 6/18 with no relative afferent pupillary defect. Anterior segment examination was unremarkable. Right fundus showed haemorrhagic retinitis along inferotemporal vessel with presence of vitreous condensation (Figure 1). CMV retinitis was confirmed by positive polymerase chain reaction (PCR) from vitreous fluid. Vitreous PCR for Herpes simplex 1 &amp; 2 and Varicella zoster were not detected. HIV serology was negative and she was not diabetic. Her absolute lymphocyte count was 0.67 x 109/L. The patient was started on daily intravenous Ganciclovir 5mg/kg BD for two weeks and biweekly intravitreal Ganciclovir 2mg/0.1 ml. Azathioprine was withheld in view that the retinitis was most probably due to immunosuppressive state. Oral prednisolone was reduced to 10mg OD. Her vision improved to 6/9 and lesions resolved (Figure 2). Absolute neutrophil count at the end of antiviral therapy was 2.60 x 109/L while absolute lymphocyte count increased to 1.10 x 109/L. Due to financial reasons and non-availability of oral valganciclovir in our hospital, patient was not able to be started on the maintenance Ganciclovir, nevertheless intravitreal Ganciclovir was continuously given as biweekly till the lesion become scarred. At week 3 when the retinitis became inactive, the intravitreal Ganciclovir was stopped.&nbsp;</div>\r\n<div>\r\n	After a month without Azathioprine, her systemic disease flared up. She had severe generalized skin itchiness and redness, Heliotrope rash, Gottron papules and proximal myopathy. Oral Azathioprine 50mg OD was recommenced and skin condition was controlled.&nbsp;</div>\r\n<div>\r\n	A month later, right eye CMV retinitis was reactivated with the lesion approaching the macula (Figure 3). Azathioprine was withheld again. She was treated with biweekly intravitreal Ganciclovir for four weeks. The retinitis subsequently resolved with 6/9 vision (Figure 4). Skin condition was otherwise status quo. The patient opted to discontinue azathioprine in fear that the retinitis would recur. Contralateral eye was not involved throughout the follow-up.&nbsp;</div>\r\n<div>\r\n	Three months later, the patient developed pathological lumbar fracture. A magnetic resonance imaging of spine incidentally revealed a lung mass. The patient refused for lung biopsy and was diagnosed with presumed stage IV lung malignancy with bone metastasis. She declined chemotherapy and immunosuppressant; and defaulted all follow up. According to the phone interview, her ocular symptoms did not recur. She passed away ten months after the diagnosis of lung malignancy.</div>\r\n<div>\r\n	Discussion</div>\r\n<div>\r\n	CMV retinitis was well recognized in patients receiving immunosuppressant. This is evidenced in a study, where CMV antigenemia was detected in 60% of patients taking prednisolone of more than 30 mg/day, suggesting that CMV infection occurs with high frequency during immunosuppressive therapy (Yoshihara et al. 1999). In patients with inflammatory connective tissue disease, CMV DNA which was previously not present prior to immunosuppressive therapy, was detected in peripheral blood leukocytes in 70% of patients after receiving immunosuppressant. This signifies a high risk of CMV reactivation in patients requiring immunosuppression (Mori et al. 2004). In this case, we believed that recommencement of Azathioprine after the first course of systemic Ganciclovir promoted the reactivation of CMV infection.&nbsp;</div>\r\n<div>\r\n	Another reason contributing the recurrence was the patient might already have lung malignancy during the event of CMV retinitis. The chronicity of the advance malignancy as evident by bone metastasis could further suppress the patient&rsquo;s immune state. To date, CMV retinitis was reported in patients with leukemia, lymphoma, and multiple myeloma during or after receiving maintenance therapy (Han et al. 2015; Downes et al. 2016). Only one case was reported in breast malignancy (Downes et al. 2016). However, incidence of CMV retinitis in lung malignancy, or in malignancy prior to chemotherapy has not been previously reported.&nbsp;</div>\r\n<div>\r\n	Although dermatomyositis itself can compromise host immunity, it is less likely in this case because the disease activity of dermatomyositis was not significant at the time of diagnosis of both CMV retinitis and its reactivation (Ku et al. 1999). CMV retinitis did not recur despite the patient was no longer on immunosuppressant thereafter.&nbsp;</div>\r\n<div>\r\n	This patient was given an initial &lsquo;induction&rsquo; course of systemic Ganciclovir for 14 days to inactivate CMV while preventing systemic spread and further enlargement of retinal lesions. Ideally, maintenance treatment with oral Valganciclovir is the best option following induction course. Moreover it would be expected for the patient to develop recurrent CMV retinitis when azathioprine was reinstituted. However, oral Valganciclovir is too costly, therefore she chose to have intravitreal Ganciclovir whenever disease gets reactivated. Another reason intravitreal Ganciclovir chosen as alternate treatment modality for reactivation was to avoid any possible systemic effect it may have on the bone marrow. This is because systemic Ganciclovir therapy can cause myelosuppression, hence an already compromised immune system may be further suppressed (Langner-Wegscheider et al. 2010). Fortunately, our patient showed a quick response to Ganciclovir in both events. In cases of resistance, other choices of treatment include Foscarnet and Cidofovir, however all of which are virustatic (Au Eong et al. 1999).</div>\r\n<div>\r\n	Visual outcome in this patient was promising as the CMV retinitis was detected early. Although maintenance treatment could not be given, close monitoring and intensive intravitreal injection were able to secure her vision. At times, vision remains the main concern of the patient, as the patient was willing to discontinue Azathioprine for the fear of recurrence of CMV retinitis.&nbsp;</div>\r\n<div>\r\n	Conclusion</div>\r\n<div>\r\n	It is a great challenge for both ophthalmologists and rheumatologists to treat concurrent CMV retinitis and dermatomyositis. As the damage to the retina caused by CMV retinitis is permanent, early detection of the disease remains critical in preserving the vision. Patients on immunosuppressant have to be warned of risk of CMV retinitis and should be educated about reporting visual disturbances (floaters, blurred vision, or scotoma). Close follow-up by ophthalmologists is at utmost importance because CMV retinitis usually occurs in periphery retina and patients might remain asymptomatic. Co-management with rheumatologists is essential for monitoring of the systemic disease, adjustment of immunosuppressant and malignancy screening in dermatomyositis.&nbsp;</div>\r\n', 1),
(67727, 67713, '<div>\r\n	QUESTION</div>\r\n<div>\r\n	A 30-year Malay gentleman (South-East Asian) ethnicity has been suffering from a left facial tumour, progressively increasing in size since birth. The tumour exits from the left orbital cavity and has displaced his left eye globe inferior posteriorly (Figure 1). Left eye vision has been progressively deteriorating in his left eye with the ability to only perceive bright light. He is of a short stature with multiple facial nodules. Spot the diagnosis and what other symptoms the patient might exhibit?&nbsp;</div>\r\n<div>\r\n	&nbsp;</div>\r\n<div>\r\n	Mail your response to the editorial address (medhealthukm@gmail.com). Answers</div>\r\n<div>\r\n	will be published in the 2017(1) issue of Medicine and Health Journal.</div>\r\n', 1),
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-- --------------------------------------------------------

--
-- Table structure for table `content_field_left_related`
--

CREATE TABLE IF NOT EXISTS `content_field_left_related` (
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  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_left_related_nid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_left_related_nid` (`field_left_related_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_field_links`
--

CREATE TABLE IF NOT EXISTS `content_field_links` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_links_url` varchar(2048) DEFAULT NULL,
  `field_links_title` varchar(255) DEFAULT NULL,
  `field_links_attributes` mediumtext,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_field_main_image`
--

CREATE TABLE IF NOT EXISTS `content_field_main_image` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_main_image_fid` int(11) DEFAULT NULL,
  `field_main_image_list` tinyint(4) DEFAULT NULL,
  `field_main_image_data` text,
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_main_image`
--

INSERT INTO `content_field_main_image` (`vid`, `nid`, `field_main_image_fid`, `field_main_image_list`, `field_main_image_data`, `delta`) VALUES
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(41, 38, NULL, NULL, NULL, 0),
(346, 343, 397, 1, 'a:2:{s:5:"title";s:146:" Figure 1. Correlation between the superior quadrant retinal nerve fibre layer (RNFL) and Central corneal thickness in Primary Open Angle Glaucoma";s:3:"alt";s:0:"";}', 0),
(346, 343, 398, 1, 'a:2:{s:5:"title";s:148:" Figure  2.  Correlation  between  the  average(mean) retinal nerve fiber layer (RNFL) and Central corneal thickness in Primary Open Angle Glaucoma ";s:3:"alt";s:0:"";}', 1),
(346, 343, 458, 1, 'a:2:{s:5:"title";s:140:"Table 1. Intraocular Pressure and Central Corneal Thickness for the normal tension glaucoma, primary open angle glaucoma and control group. ";s:3:"alt";s:0:"";}', 2),
(346, 343, 459, 1, 'a:2:{s:5:"title";s:79:" Table 2: Mean retinal nerve fiber layer thickness measurement in all quadrant ";s:3:"alt";s:0:"";}', 3),
(346, 343, 460, 1, 'a:2:{s:5:"title";s:85:"Table 3: Correlation between central corneal thickness and retinal nerve fibre layer ";s:3:"alt";s:0:"";}', 4),
(353, 350, 400, 1, 'a:2:{s:5:"title";s:72:"Figure.1: CT scan shows a cystic mass in the left adnexa (white arrow). ";s:3:"alt";s:0:"";}', 0),
(353, 350, 401, 1, 'a:2:{s:5:"title";s:8:"Figure 2";s:3:"alt";s:0:"";}', 1),
(360, 357, 404, 1, 'a:2:{s:5:"title";s:195:"Figure  1:  Peripheral  blood  film  of  the  patient  showed  presence  of  numerous  blast  cells  with  high nuclear:cytoplasmic ratio and inconspicuous nucleoli (arrow). (Wright stain, x 200)";s:3:"alt";s:0:"";}', 0),
(360, 357, 405, 1, 'a:2:{s:5:"title";s:183:"Figure 2: Flow cytometric analysis of the bone marrow sample showed a single population of blast cells (red) expressing CD 3, CD 7, CD 4 and CD8 but negative for CD 34 and B-markers. ";s:3:"alt";s:0:"";}', 1),
(368, 365, NULL, NULL, NULL, 0),
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(369, 366, 466, 1, 'a:2:{s:5:"title";s:82:"TABLE 1. Patient’s characteristics and results of cytogenetics, FISH and RT-PCR.";s:3:"alt";s:0:"";}', 1),
(369, 366, 467, 1, 'a:2:{s:5:"title";s:67:"TABLE 2.  Correlation between Conventional Cytogenetics and RT-PCR.";s:3:"alt";s:0:"";}', 2),
(369, 366, 468, 1, 'a:2:{s:5:"title";s:46:"TABLE 3.  Correlation between FISH and RT-PCR.";s:3:"alt";s:0:"";}', 3),
(369, 366, 469, 1, 'a:2:{s:5:"title";s:65:"TABLE 4.  Correlation between Conventional Cytogenetics and FISH.";s:3:"alt";s:0:"";}', 4),
(370, 367, 471, 1, 'a:2:{s:5:"title";s:76:"Figure 1:  A  Conceptual  Framework  -  NURSQUAL consisting of four domains ";s:3:"alt";s:0:"";}', 0),
(370, 367, 472, 1, 'a:2:{s:5:"title";s:94:" Figure 2:  Questions  related  to  element  of  technical competence and information giving  ";s:3:"alt";s:0:"";}', 1),
(370, 367, 473, 1, 'a:2:{s:5:"title";s:55:"Figure 2A: Questions related to element of assurance.  ";s:3:"alt";s:0:"";}', 2),
(370, 367, 474, 1, 'a:2:{s:5:"title";s:52:"Figure 2B: Questions related to element of empathy  ";s:3:"alt";s:0:"";}', 3),
(370, 367, 475, 1, 'a:2:{s:5:"title";s:57:"Table 1:  Respondents’ demographic data distribution.  ";s:3:"alt";s:0:"";}', 4),
(370, 367, 476, 1, 'a:2:{s:5:"title";s:121:"Table 2:  Respondent’s  satisfaction  score  with technical  competence,  information  giving, assurance and empathy.  ";s:3:"alt";s:0:"";}', 5),
(370, 367, 477, 1, 'a:2:{s:5:"title";s:172:"Table  3:  Pearson  correlation  coefficient:  correlation between  technical  competence,  information giving,  assurance  and  empathy  with respondent’s satisfaction  ";s:3:"alt";s:0:"";}', 6),
(370, 367, 478, 1, 'a:2:{s:5:"title";s:136:"Table  4:  Prediction  of  patient  satisfaction  in  relation with  technical  competence,  information giving, assurance and empathy. ";s:3:"alt";s:0:"";}', 7),
(371, 368, 480, 1, 'a:2:{s:5:"title";s:201:"Table 1:  Demographic  data,  duration  of anaesthesia,  volume  of  fluids infused and type of cases. Values expressed  as  mean  ±  standard deviation  (SD)  and  number  (n) where appropriate.     ";s:3:"alt";s:0:"";}', 0),
(371, 368, 481, 1, 'a:2:{s:5:"title";s:161:" Table 2:  Mean  blood  glucose  levels  at different  time of anaesthesia  for two groups. Values expressed as mean  ± SD, and number  (n) where appropriate.  ";s:3:"alt";s:0:"";}', 1),
(372, 369, 483, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 0),
(372, 369, 484, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 1),
(373, 370, 486, 1, 'a:2:{s:5:"title";s:97:"Table 1: Characteristics of socio-demographic variables for newly diagnosed breast cancer (n=100)";s:3:"alt";s:0:"";}', 0),
(373, 370, 487, 1, 'a:2:{s:5:"title";s:70:"Table 2: The Thurstone Scale Analysis on the Nine Information (n=100) ";s:3:"alt";s:0:"";}', 1),
(373, 370, 488, 1, 'a:2:{s:5:"title";s:133:"Table 3: Scales Value for the item Cure and for each Ethnic Group, Educational level, Age and Social class on the Information on Cure";s:3:"alt";s:0:"";}', 2),
(373, 370, 489, 1, 'a:2:{s:5:"title";s:124:"Table 4: Analysis of Variance (ANOVA) on information on Cure for the three ethnic groups, Educational level and Social class";s:3:"alt";s:0:"";}', 3),
(374, 371, 490, 1, 'a:2:{s:5:"title";s:113:"Table 1:  Summary of results of cytogenetic analysis, fragile site detection and PCR analysis of the FRAXA locus ";s:3:"alt";s:0:"";}', 0),
(374, 371, 491, 1, 'a:2:{s:5:"title";s:125:"Figure 1: Partial karyotype of G-banded (left) and stained (right) fragile X chromosome  (arrow indicating the fragile sites)";s:3:"alt";s:0:"";}', 1),
(374, 371, 492, 1, 'a:2:{s:5:"title";s:280:"Figure 2: Gel electrophoresis showing Polymerase chain reaction (PCR) product of FRAXA locus. Lane 1 (light band) is normal female control (TC), lanes 2 to 6 are patients’ samples (T1, T2, T3, T4, and T5 respectively). Lane 7 is a negative control (NC).M is the100bp DNA ladder ";s:3:"alt";s:0:"";}', 2),
(375, 372, 494, 1, 'a:2:{s:5:"title";s:104:"TABLE 1: Results of Conventional and FISH analysis using locus specific probe of the nine cases studied ";s:3:"alt";s:0:"";}', 0),
(375, 372, 495, 1, 'a:2:{s:5:"title";s:259:"FIGURE 1: Giemsa-banded chromosomes showing the karyotype of the patient with DiGeorge syndrome (case 9).  The whole of the long arm of chromosome 22 is translocated onto the short arm of chromosome 9, resulting in monosomy of chromosome 22 (bottom arrow).   ";s:3:"alt";s:0:"";}', 1),
(375, 372, 496, 1, 'a:2:{s:5:"title";s:213:"FIGURE 2:  Interphase  and  metaphase  spreads  hybridised  with  LSI  DiGeorge  TUPLE  1 region  probe.  Absence  of the  red  signal  on  one  of  the  chromosome  22 indicates deletion of the locus at 22q11.2. ";s:3:"alt";s:0:"";}', 2),
(375, 372, 498, 1, 'a:2:{s:5:"title";s:188:"FIGURE 3: Giemsa-banded chromosomes showing karyotype of patient’s  (Case 9)  mother with translocation between the long arm of chromosome 22 and the short arm of chromosome 9 (arrows). ";s:3:"alt";s:0:"";}', 3),
(376, 373, 500, 1, 'a:2:{s:5:"title";s:79:"TABLE 1: Laboratory findings in two cases of Biphenotypic Acute Leukemia (BAL) ";s:3:"alt";s:0:"";}', 0),
(376, 373, 501, 1, 'a:2:{s:5:"title";s:154:"FIGURE 1:  Bone marrow aspiration of case 1 showing a heterogenous population of large  (red arrow) and small (blue arrow) blasts cells. (MGG stain x 400)";s:3:"alt";s:0:"";}', 1),
(376, 373, 502, 1, 'a:2:{s:5:"title";s:152:"FIGURE 2: Bone marrow aspiration of case 2 showing a heterogenous population of large  (red arrow) and small (blue arrow) blast cells. (MGG stain x 400)";s:3:"alt";s:0:"";}', 2),
(376, 373, 503, 1, 'a:2:{s:5:"title";s:146:"FIGURE 3: Immunophenotyping of case 1 showed co-expression CD 19 and CD33, CD  13 and CD10, HLA DR and CD34,  intra22 and intraMPO of gated cells.";s:3:"alt";s:0:"";}', 3),
(376, 373, 504, 1, 'a:2:{s:5:"title";s:72:"FIGURE 4: FISH analysis of case 2 showed presence of BCR-ABL fusion gene";s:3:"alt";s:0:"";}', 4),
(376, 373, 505, 1, 'a:2:{s:5:"title";s:91:"FIGURE 5: Molecular analysis by RT-PCR of case 2 showed presence of major BCR-ABL fragment.";s:3:"alt";s:0:"";}', 5),
(377, 374, 506, 1, 'a:2:{s:5:"title";s:83:"FIGURE 1: An axial HRCT from case 2 shows opacity of the lumen of the left cochlea.";s:3:"alt";s:0:"";}', 0),
(377, 374, 507, 1, 'a:2:{s:5:"title";s:180:"FIGURE 2:  An axial T2-weighted  MRI (case 2)  shows hypointensity  of the lumen  of the cochlea  bilaterally,  compared  to  cerebrospinal  fluid  (CSF),  which  is hyperintense. ";s:3:"alt";s:0:"";}', 1),
(377, 374, 508, 1, 'a:2:{s:5:"title";s:113:"FIGURE 3:  A coronal  T2-weighted MRI  (case  2)  shows hypointensity of the labyrinthine structures bilaterally.";s:3:"alt";s:0:"";}', 2),
(377, 374, 509, 1, 'a:2:{s:5:"title";s:108:"FIGURE 4:  A  modified  Stenver’s  (cochlear)  view  shows  partial  insertion  of  electrodes  in case 3.";s:3:"alt";s:0:"";}', 3),
(378, 375, 511, 1, 'a:2:{s:5:"title";s:234:"FIGURE 1: Histological section of laryngeal papillommatosis (Case 1) (Haematoxylin & eosin, a, x20). Cells containing HPV 6 DNA by in situ hybridisation, indicated by the dark-stained nuclei of the superficial cells (b, x40 & c, x400)";s:3:"alt";s:0:"";}', 0),
(378, 375, 512, 1, 'a:2:{s:5:"title";s:238:"FIGURE 2:	Layngeal papillomatosis. Histological section from Case 1 showing nuclear staining for p53 protein in the cells within the basal layer, and a few positive cells in the upper epithelial cells. (Immunoperoxidase, a, x 40; b, x400)";s:3:"alt";s:0:"";}', 1),
(378, 375, 513, 1, 'a:2:{s:5:"title";s:189:"FIGURE 3:	Laryngeal papillomatosis. Histological section from Case 1 showing nearly diffuse nuclear staining of p53 protein throughout the epithelial thickness (Immunoperoxidase stain, x20)";s:3:"alt";s:0:"";}', 2),
(379, 376, 514, 1, 'a:2:{s:5:"title";s:184:"FIGURE 1: FNA of paraaortic lymph node showing cells displaying abundant cytoplasm with fine vacuolations and eccentric nucleus. May-Grunwald Giemsa stain, original magnification x 200";s:3:"alt";s:0:"";}', 0),
(379, 376, 515, 1, 'a:2:{s:5:"title";s:132:"FIGURE 2: FNA smear showing neoplastic cells displaying granular cytoplasm. May- Grunwald Giemsa stain, original magnification x 200";s:3:"alt";s:0:"";}', 1),
(379, 376, 516, 1, 'a:2:{s:5:"title";s:183:"FIGURE 3: Histological section of lymph node showing sheets of malignant cells separated by fine fibrous septae, displaying clear cytoplasm in areas. H&E, original magnification x 100";s:3:"alt";s:0:"";}', 2),
(380, 377, 517, 1, 'a:2:{s:5:"title";s:273:"Figure 1(A)is the peripheral blood film of this patient before splenectomy showing numerous tear-drop poikilocytes as compared to 1(B) which is the peripheral blood film taken after splenectomy demonstrating marked reduction in tear-drop poikilocytes. (Wright stain, x 400)";s:3:"alt";s:0:"";}', 0),
(381, 378, 521, 1, 'a:2:{s:5:"title";s:99:"Figure 1: Exophytic tumour composed of villous and papillary architecture (H&E, 10x  magnification)";s:3:"alt";s:0:"";}', 0),
(381, 378, 522, 1, 'a:2:{s:5:"title";s:148:"Figure 2.  Histological section of the right obturator lymph node showing infiltration by  metastatic adenocarcinoma (H&E stain, 20x magnification).";s:3:"alt";s:0:"";}', 1),
(381, 378, 523, 1, 'a:2:{s:5:"title";s:126:"Table 1: Clinicopathological features of reported cases of villoglandular papillary  adenocarcinoma with lymph node metastasis";s:3:"alt";s:0:"";}', 2),
(382, 379, 525, 1, 'a:2:{s:5:"title";s:176:"FIGURE 1:	Case 1. Fundus photograph of the right eye showing phlebitis of the superotemporal vein with three adjacent choroidal lesions with macular oedema and disc hyperaemia.";s:3:"alt";s:0:"";}', 0),
(382, 379, 526, 1, 'a:2:{s:5:"title";s:146:"FIGURE 2:	Case 2. Fundus photograph of the right eye showing occlusive vasculitis involving the infero-temporal arteriole with adjacent retinitis.";s:3:"alt";s:0:"";}', 1),
(383, 380, 952, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(383, 380, 953, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(383, 380, 954, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(384, 381, 529, 1, 'a:2:{s:5:"title";s:59:"Figure 1 : Respondents before and after PCL anxiety levels.";s:3:"alt";s:0:"";}', 0),
(384, 381, 530, 1, 'a:2:{s:5:"title";s:62:"Figure 2 : Respondents before and after PCI depression levels.";s:3:"alt";s:0:"";}', 1),
(384, 381, 531, 1, 'a:2:{s:5:"title";s:71:"Table 1: Respondent before and after PCI anxiety and depression levels.";s:3:"alt";s:0:"";}', 2),
(384, 381, 532, 1, 'a:2:{s:5:"title";s:47:"Table 2 : Respondents’ socio demographic data";s:3:"alt";s:0:"";}', 3),
(384, 381, 533, 1, 'a:2:{s:5:"title";s:95:"Table 2A : Respondents’ demographic data in relation to before and after PCI, anxiety levels ";s:3:"alt";s:0:"";}', 4),
(384, 381, 534, 1, 'a:2:{s:5:"title";s:104:"Table 2B : Respondents’ socio demographic data in relation to before and after PCI,  depression levels";s:3:"alt";s:0:"";}', 5),
(385, 382, 536, 1, 'a:2:{s:5:"title";s:54:"Table 1: 	Socio demographic data of respondents (n=38)";s:3:"alt";s:0:"";}', 0),
(385, 382, 537, 1, 'a:2:{s:5:"title";s:96:"Table 2:	Distribution of level of anxiety and depression among high risk pregnant mothers (n=38)";s:3:"alt";s:0:"";}', 1),
(385, 382, 538, 1, 'a:2:{s:5:"title";s:91:"Table 3:	Questionnaires related to Factors Influencing the level of anxiety and depression.";s:3:"alt";s:0:"";}', 2),
(386, 383, 541, 1, 'a:2:{s:5:"title";s:66:"Table I:  	Sociodemographic characteristic of the adolescent girls";s:3:"alt";s:0:"";}', 0),
(386, 383, 542, 1, 'a:2:{s:5:"title";s:96:"Table II:	Association between dysmenorrhoea with socio-demographic and menstrual characteristics";s:3:"alt";s:0:"";}', 1),
(386, 383, 543, 1, 'a:2:{s:5:"title";s:75:"Table III:  	Severity of dysmenorrhoeic pain with school and class absences";s:3:"alt";s:0:"";}', 2),
(386, 383, 544, 1, 'a:2:{s:5:"title";s:116:"Table IV:	Mean pain score with perceived concentration and sports participation among adolescents with dysmenorrhoea";s:3:"alt";s:0:"";}', 3),
(387, 384, 546, 1, 'a:2:{s:5:"title";s:23:"Table 1.	Domain Scoring";s:3:"alt";s:0:"";}', 0),
(387, 384, 547, 1, 'a:2:{s:5:"title";s:72:"Table 2:	Socio-demographic and marital characteristics of the respondent";s:3:"alt";s:0:"";}', 1),
(387, 384, 548, 1, 'a:2:{s:5:"title";s:42:"Figure 1: 	Frequency of sexual intercourse";s:3:"alt";s:0:"";}', 2),
(387, 384, 549, 1, 'a:2:{s:5:"title";s:62:"Table 3: 	Frequency of sexual intercourse among women with FSD";s:3:"alt";s:0:"";}', 3),
(387, 384, 550, 1, 'a:2:{s:5:"title";s:94:"Table 4:	Domains of sexual functioning among groups of women with low and high frequency of SI";s:3:"alt";s:0:"";}', 4),
(388, 385, 552, 1, 'a:2:{s:5:"title";s:32:"Table 1. Results of FNA cytology";s:3:"alt";s:0:"";}', 0),
(388, 385, 558, 1, 'a:2:{s:5:"title";s:77:"Table 2. Results of FNA cytology with subsequent histopathological diagnosis.";s:3:"alt";s:0:"";}', 1),
(388, 385, 554, 1, 'a:2:{s:5:"title";s:72:"Table 3.	Summary of cyto-histopathological cor-relation of all 361 cases";s:3:"alt";s:0:"";}', 2),
(388, 385, 555, 1, 'a:2:{s:5:"title";s:111:"Table 4.	Calculations of diagnostic accuracy, sensitivity, specificity, negative and positive predictive values";s:3:"alt";s:0:"";}', 3),
(388, 385, 556, 1, 'a:2:{s:5:"title";s:120:"Figure 1: 	Papillary carcinoma. Pale and powdery nuclear chromatin with longitudinal grooves. (Papanicolaou stain, 400X)";s:3:"alt";s:0:"";}', 4),
(388, 385, 557, 1, 'a:2:{s:5:"title";s:95:"Figure 2: 	Follicular neoplasm displaying coarse chromatin pattern. (Papanicolaou stain, 400 X)";s:3:"alt";s:0:"";}', 5),
(389, 386, 560, 1, 'a:2:{s:5:"title";s:37:"Table 1 : Sociodemographics variables";s:3:"alt";s:0:"";}', 0),
(389, 386, 561, 1, 'a:2:{s:5:"title";s:65:"Table 2 : Uterine measurements by menopausal status and ethnicity";s:3:"alt";s:0:"";}', 1),
(389, 386, 562, 1, 'a:2:{s:5:"title";s:78:"Table 3 : Prevalence and size of fibromyoma by menopausal status and ethnicity";s:3:"alt";s:0:"";}', 2),
(389, 386, 563, 1, 'a:2:{s:5:"title";s:65:"Table 4: Endometrial thickness by menopausal status and ethnicity";s:3:"alt";s:0:"";}', 3),
(389, 386, 564, 1, 'a:2:{s:5:"title";s:59:"Table 5 : Ovarian volume by menopausal status and ethnicity";s:3:"alt";s:0:"";}', 4),
(389, 386, 565, 1, 'a:2:{s:5:"title";s:91:"Table 6 : Ovarian cyst prevalence, dimensions and volume by menopausal status and ethnicity";s:3:"alt";s:0:"";}', 5),
(389, 386, 566, 1, 'a:2:{s:5:"title";s:74:"Figure 1: Uterine size (normogram) in pre and postmenopausal women by age.";s:3:"alt";s:0:"";}', 6),
(389, 386, 567, 1, 'a:2:{s:5:"title";s:84:"Figure 2: Endometrial thickness (normogram) of pre and postmenopausal women by age. ";s:3:"alt";s:0:"";}', 7),
(389, 386, 569, 1, 'a:2:{s:5:"title";s:75:"Figure 3: Ovarian volume (normogram) of pre and postmenopausal women by age";s:3:"alt";s:0:"";}', 8),
(390, 387, 571, 1, 'a:2:{s:5:"title";s:54:"Table I: 	Smoking habits of the respondents who smoked";s:3:"alt";s:0:"";}', 0),
(390, 387, 572, 1, 'a:2:{s:5:"title";s:28:"Table 2: Reasons for smoking";s:3:"alt";s:0:"";}', 1),
(391, 388, 573, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(391, 388, 574, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(391, 388, 575, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(391, 388, 576, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
(391, 388, 577, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 4),
(392, 389, 579, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(392, 389, 580, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(392, 389, 581, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(392, 389, 582, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(392, 389, 583, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 4),
(393, 390, 585, 1, 'a:2:{s:5:"title";s:201:"Fig. 1A: 	Low power view of section of right parotid mass showing dense eosinophilic infiltration within the nodal parenchymal and fibroadipose tissue. (x 20 magnification; Haematoxylin & Eosin stain).";s:3:"alt";s:0:"";}', 0),
(393, 390, 586, 1, 'a:2:{s:5:"title";s:183:"Fig. 1B: Higher magnification of Fig. 1A showing infiltration by eosinophils displaying orangeophilic cytoplasm and bilobed nucleus. (x 100 magnification; Haematoxylin & Eosin stain).";s:3:"alt";s:0:"";}', 1),
(394, 391, 589, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(394, 391, 594, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(394, 391, 591, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(394, 391, 592, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(394, 391, 593, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 4),
(395, 392, 595, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(395, 392, 596, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(395, 392, 597, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(396, 393, 599, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(396, 393, 600, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(396, 393, 605, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(396, 393, 601, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(396, 393, 602, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 4),
(396, 393, 603, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 5),
(397, 394, 606, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(397, 394, 607, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(397, 394, 608, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(397, 394, 609, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
(398, 395, 611, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(398, 395, 612, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(399, 396, 615, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(399, 396, 616, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(399, 396, 617, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(399, 396, 618, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(399, 396, 619, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 4),
(400, 397, 621, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 0),
(400, 397, 622, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 1),
(401, 398, NULL, NULL, NULL, 0),
(402, 399, 625, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(402, 399, 626, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(402, 399, 627, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(402, 399, 628, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
(403, 400, 629, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(403, 400, 630, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(404, 401, 632, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 0),
(405, 402, 634, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(405, 402, 635, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(405, 402, 636, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(406, 403, 638, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(406, 403, 639, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(406, 403, 640, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(406, 403, 641, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(406, 403, 642, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 4),
(407, 404, 645, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(407, 404, 646, 1, 'a:2:{s:5:"title";s:127:"Figure 3:  FISH analysis of patient TS6 showing interphase cells and metaphase spread with SRY gene (red signals, white arrows)";s:3:"alt";s:0:"";}', 1),
(407, 404, 647, 1, 'a:2:{s:5:"title";s:127:"Figure 4: 	FISH result in patient TS6 showing translocation of the SRY gene onto one of the autosomes (red signal, white arrow)";s:3:"alt";s:0:"";}', 2),
(407, 404, 648, 1, 'a:2:{s:5:"title";s:260:"Figure 5: 	FISH result of patient TS7 showing two interphase cells with one cell displaying 45, X  only (one green signal) and another cell with an extra and intense green signal, consistent with an additional normal cell line 45XX ( green signal, white arrow)";s:3:"alt";s:0:"";}', 3),
(408, 405, 649, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(408, 405, 650, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(408, 405, 651, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(408, 405, 652, 1, 'a:2:{s:5:"title";s:55:"Figure 1:	Main sources of information on breastfeeding.";s:3:"alt";s:0:"";}', 3),
(409, 406, 654, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(409, 406, 655, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(409, 406, 656, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(409, 406, 657, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(409, 406, 658, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 4),
(409, 406, 659, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 5),
(409, 406, 660, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 6),
(410, 407, 663, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(410, 407, 664, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(410, 407, 665, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(410, 407, 666, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
(410, 407, 667, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 4),
(411, 408, 668, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(411, 408, 669, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(412, 409, 671, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(412, 409, 672, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(412, 409, 673, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(413, 410, 675, 1, 'a:2:{s:5:"title";s:158:"Figure 1:	Ultrasound showed a fairly well defined inhomogenous soft tissue mass, measuring about 4x4x2.6cm in the deep subcutaneous region in left iliac fossa";s:3:"alt";s:0:"";}', 0),
(413, 410, 676, 1, 'a:2:{s:5:"title";s:393:"Figure 2.:	(A) Histologic examination showed the tumour mass is surrounded by fibrous tissue and adipocytes (Haematoxylin and Eosin, magnification x100). (B) These cells are large exhibiting pleomorphic and vesicular nuclei and abundant cytoplasm. Some of the nuclei are kidney in shaped Anaplastic large cell lymphoma showing large cells with eccentric kidney shape nuclei [(↑) H & E, x400]";s:3:"alt";s:0:"";}', 1),
(413, 410, 677, 1, 'a:2:{s:5:"title";s:189:"Figure 3:	FISH showing nuclei with three signals are positive with the rearrangement (thick arrows) while nuclei with two fusion signal pattern lack the ALK gene rearrangement (thin arrows)";s:3:"alt";s:0:"";}', 2),
(413, 410, 678, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
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(462, 459, 919, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(462, 459, 920, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(462, 459, 921, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 2),
(463, 460, 923, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(463, 460, 924, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(463, 460, 925, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(463, 460, 926, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 3),
(464, 461, 928, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 0),
(464, 461, 929, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 1),
(464, 461, 930, 1, 'a:2:{s:5:"title";s:0:"";s:3:"alt";s:0:"";}', 2),
(464, 461, 931, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}', 3),
(465, 462, 934, 1, 'a:2:{s:5:"title";s:149:"Figure 1: Ultrasound of the left testis which demonstrates an increase in doppler signal within the testis. The left testis is in the inguinal canal.";s:3:"alt";s:0:"";}', 0),
(465, 462, 935, 1, 'a:2:{s:5:"title";s:275:"Figure 2: Voiding phase of micturating cystourethrogram (MCU) in left anterior oblique (LAO) view which shows bulbous dilatation of the posterior urethra (white arrows) with transverse linear filling defect distal to it representing a posterior urethral valve (black arrow). ";s:3:"alt";s:0:"";}', 1),
(466, 463, NULL, NULL, NULL, 0),
(467, 464, NULL, NULL, NULL, 0),
(468, 465, NULL, NULL, NULL, 0),
(470, 467, 451, 1, 'a:2:{s:5:"title";s:64:"Figure 1: Clinical pathway for patient attended the CSRC program";s:3:"alt";s:0:"";}', 0),
(470, 467, 452, 1, 'a:2:{s:5:"title";s:82:"Figure 2: Types of rehabilitation intervention received by stroke patients in CSRC";s:3:"alt";s:0:"";}', 1),
(470, 467, 456, 1, 'a:2:{s:5:"title";s:109:" Table 1: Demographic and clinical profile of stroke patients attending Combined Stroke Rehabilitation Clinic";s:3:"alt";s:0:"";}', 2),
(470, 467, 457, 1, 'a:2:{s:5:"title";s:96:"Table 2: Progress of patients over time based on Modified Barthel Index and Berg Balance Score  ";s:3:"alt";s:0:"";}', 3),
(471, 468, NULL, NULL, NULL, 0),
(472, 469, NULL, NULL, NULL, 0),
(475, 472, NULL, NULL, NULL, 0),
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(920, 915, 1032, 1, 'a:2:{s:5:"title";s:111:"Figure 1: A) ‘Double-I’ construct. B) new improved double ‘J’ with limb immobilization extenders (LIE).";s:3:"alt";s:0:"";}', 0),
(920, 915, 1033, 1, 'a:2:{s:5:"title";s:255:"Figure 2: Six pressure sensors location at I:Left, II. Middle, III: Right. A- Left Pelvis (above); B- Left Pelvis (below); C- Right pelvis (above); D- Right pelvis (below); E- Posterior pelvis- sacral area (above); F- Posterior pelvis- sacral area (below)";s:3:"alt";s:0:"";}', 1),
(920, 915, 1034, 1, 'a:2:{s:5:"title";s:91:"Figure 3: The new construct ‘Double J’ design, A-anterior aspect with sleeve (arrowed) ";s:3:"alt";s:0:"";}', 2),
(920, 915, 1035, 1, 'a:2:{s:5:"title";s:105:"Figure 3:B-the posterior aspect (Black arrows shows flappable area and blue arrow the essential GT belt).";s:3:"alt";s:0:"";}', 3),
(920, 915, 1036, 1, 'a:2:{s:5:"title";s:56:"Figure 4: Pressure measurements on left pelvis for 50 se";s:3:"alt";s:0:"";}', 4),
(920, 915, 1037, 1, 'a:2:{s:5:"title";s:62:"Figure 5: Pressure measurements on right pelvis for 50 seconds";s:3:"alt";s:0:"";}', 5),
(920, 915, 1039, 1, 'a:2:{s:5:"title";s:63:"Figure 6: Pressure measurements on middle pelvis for 50 seconds";s:3:"alt";s:0:"";}', 6),
(920, 915, 1040, 1, 'a:2:{s:5:"title";s:130:"Figure 7: A) Open book pelvic fracture. B) Radiolucent property of BRIM and the ability to close/reduced the pelvic volume tested.";s:3:"alt";s:0:"";}', 7),
(929, 924, 1044, 1, 'a:2:{s:5:"title";s:120:"Figure 1: Proportion of respondents who were satisfied or not satisfied according to the domains of patient satisfaction";s:3:"alt";s:0:"";}', 0),
(933, 928, 1046, 1, 'a:2:{s:5:"title";s:85:"Figure 1: Illustration of the TSH receptor complex and the competitive test principle";s:3:"alt";s:0:"";}', 0),
(933, 928, 1047, 1, 'a:2:{s:5:"title";s:274:"Figure 2: The ROC curve performed on 46 GD (sensitivity cohort), 20 patients with other thyroid disease and 58 apparently normal control (specificity cohort) The optimal cut of value was calculated at 1.69 IU/l (sensitivity 94%, specificity 98%), AUC 0.99 (95% CI: 0.98-1.0)";s:3:"alt";s:0:"";}', 1),
(936, 931, 1049, 1, 'a:2:{s:5:"title";s:109:"Figure 1. Histological section of IDC showed immunopositivity for Bcl-2 in the cytoplasm (magnification x400)";s:3:"alt";s:0:"";}', 0),
(936, 931, 1050, 1, 'a:2:{s:5:"title";s:112:"Figure 2. Histological section of IDC showed strong immunopositivity for p53 in the nucleus (magnification x400)";s:3:"alt";s:0:"";}', 1),
(944, 939, NULL, NULL, NULL, 0),
(946, 941, 1053, 1, 'a:2:{s:5:"title";s:112:"Figure 1: Fundus photograph showing bilateral macula oedema with yellow subretinal deposits and dot haemorrhages";s:3:"alt";s:0:"";}', 0),
(946, 941, 1054, 1, 'a:2:{s:5:"title";s:112:"Figure 1: Fundus photograph showing bilateral macula oedema with yellow subretinal deposits and dot haemorrhages";s:3:"alt";s:0:"";}', 1),
(946, 941, 1055, 1, 'a:2:{s:5:"title";s:114:"Figure 2: Optical coherence tomography demonstrate subretinal fluid collection on both right and left eyes (arrow)";s:3:"alt";s:0:"";}', 2),
(946, 941, 1056, 1, 'a:2:{s:5:"title";s:114:"Figure 2: Optical coherence tomography demonstrate subretinal fluid collection on both right and left eyes (arrow)";s:3:"alt";s:0:"";}', 3),
(950, 945, 1058, 1, 'a:2:{s:5:"title";s:124:"Figure 1: Interphase FISH analysis of (a) FTC (FC-03) showing trisomy nuclei with three orange and two green signal pattern;";s:3:"alt";s:0:"";}', 0),
(950, 945, 1059, 1, 'a:2:{s:5:"title";s:96:"Figure 1: FTC (FC-19) showing a tetrasomy nucleus with four orange and two green signal pattern;";s:3:"alt";s:0:"";}', 1),
(950, 945, 1060, 1, 'a:2:{s:5:"title";s:111:"Figure 1:FTA (Hurthle cell type, FA-18) showing tetrasomy nuclei with four orange and two green signal pattern.";s:3:"alt";s:0:"";}', 2),
(997, 991, 1263, 1, 'a:2:{s:5:"title";s:152:"Figure 1: The DPPH scavenging activity of CA extracts. Ascorbic acid and BHT were used as positive controls. Results were expressed as means ±SD (n=6).";s:3:"alt";s:0:"";}', 0),
(997, 991, 1264, 1, 'a:2:{s:5:"title";s:333:"Figure 2: COX-1 and COX-2 enzyme inhibitory activities of CA extracts at 30 g/ml, and bioactive compounds at 5 μg/ml. Commercial NSAIDs, aspirin at 5 mg/ml concentration was tested as positive control. Results were expressed as means ± ±SD (n=4). a, b, c, d p<0.05 vs. aspirin, ethanol, methanol and aqueous extract, respectively.";s:3:"alt";s:0:"";}', 1),
(997, 991, 1265, 1, 'a:2:{s:5:"title";s:320:"Figure 3: PGE2 production of TPA-induced fibroblast cell treated with CA extracts (30μg/ml) and bioactive compounds (5 μg/ml). Aspirin was used as positive control at 5 μg/ml. Results were expressed as means ±SEM (n=6). a, b, c, d, e p<0.05 vs. control, aspirin, ethanol, methanol, and aqueous extract, respectively.";s:3:"alt";s:0:"";}', 2),
(1000, 994, NULL, NULL, NULL, 0),
(1006, 1000, 1238, 1, 'a:2:{s:5:"title";s:80:"Figure 1: Example of Absorbance Profiles for Various Combinations of Test sample";s:3:"alt";s:0:"";}', 0),
(1006, 1000, 1239, 1, 'a:2:{s:5:"title";s:94:"Figure 2: Comparison between Local and International Sunscreens in terms of SPF Label Accuracy";s:3:"alt";s:0:"";}', 1),
(1006, 1000, 1240, 1, 'a:2:{s:5:"title";s:87:"Figure 3: Comparison of Relative UVAPF Level between Local and International Sunscreens";s:3:"alt";s:0:"";}', 2),
(1006, 1000, 1241, 1, 'a:2:{s:5:"title";s:81:"Figure 4: Comparison of Photostability between Local and International Sunscreens";s:3:"alt";s:0:"";}', 3),
(1011, 1005, 1242, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 0),
(1011, 1005, 1243, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 1),
(1011, 1005, 1244, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 2),
(1011, 1005, 1245, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 3),
(1011, 1005, 1246, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 4),
(1011, 1005, 1247, 1, 'a:2:{s:5:"title";s:257:"Figure 1: Photograph of the fundus of the right (a) and left (d) eyes showing severe vasculitis and macular oedema (arrow), with corresponding macular elevation on OCT macula (c and f). Photos (b) and (e) show the scan cut at which macula elevation is seen.";s:3:"alt";s:0:"";}', 5),
(1011, 1005, 1248, 1, 'a:2:{s:5:"title";s:240:"Figure 2: Photos a and b show fluorescein angiography findings which correspond with clinical pictures in Figure 1. Dark areas represent hypoperfusion (green arrow) and hyperfluorescent area show leakages from vasculitic vessels (red arrow)";s:3:"alt";s:0:"";}', 6),
(1011, 1005, 1249, 1, 'a:2:{s:5:"title";s:240:"Figure 2: Photos a and b show fluorescein angiography findings which correspond with clinical pictures in Figure 1. Dark areas represent hypoperfusion (green arrow) and hyperfluorescent area show leakages from vasculitic vessels (red arrow)";s:3:"alt";s:0:"";}', 7),
(1014, 1008, 1251, 1, 'a:2:{s:5:"title";s:134:"Figure 1: USG revealed a dilated proximal CBD and right intra-hepatic ducts with tubular hyper-echoic structure and mild splenomegaly.";s:3:"alt";s:0:"";}', 0),
(1014, 1008, 1252, 1, 'a:2:{s:5:"title";s:106:"Figure 2: CT-abdomen revealed distal CBD obstruction with linear density, compatible with image of a worm.";s:3:"alt";s:0:"";}', 1),
(1014, 1008, 1253, 1, 'a:2:{s:5:"title";s:37:"Figure 3: ERCP revealed a worm in CBD";s:3:"alt";s:0:"";}', 2),
(1014, 1008, 1254, 1, 'a:2:{s:5:"title";s:104:"Figure 4: Helminthic infestation that was removed from the patient’s biliary tract via ERCP procedure.";s:3:"alt";s:0:"";}', 3),
(1020, 1014, NULL, NULL, NULL, 0),
(1026, 1020, 1257, 1, 'a:2:{s:5:"title";s:98:"Figure 1: Full blood picture showing severe anaemia with ovalostomatocytes. (Wright’s stain x40)";s:3:"alt";s:0:"";}', 0),
(1026, 1020, 1258, 1, 'a:2:{s:5:"title";s:116:"Figure 2: Presence of blast cell in the full blood picture. The blast show cytoplasmic blebs. (Wright’s stain x40)";s:3:"alt";s:0:"";}', 1),
(1026, 1020, 1259, 1, 'a:2:{s:5:"title";s:56:"Figure 3: Blasts with cytoplasmic blebs. (MGG stain x40)";s:3:"alt";s:0:"";}', 2),
(1026, 1020, 1260, 1, 'a:2:{s:5:"title";s:92:"Figure 4: The megakaryoblasts are negative to Myeloperoxidase stain. (Peroxidase stain, x40)";s:3:"alt";s:0:"";}', 3),
(1026, 1020, 1261, 1, 'a:2:{s:5:"title";s:73:"Figure 5: High resolution karyotyping result showing the t(1;22)(p13;q13)";s:3:"alt";s:0:"";}', 4),
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INSERT INTO `content_field_main_image` (`vid`, `nid`, `field_main_image_fid`, `field_main_image_list`, `field_main_image_data`, `delta`) VALUES
(1122, 1114, 1366, 1, 'a:2:{s:5:"title";s:87:"Figure 1: TURP sample showed granulomatous inflammation accompanied by central necrosis";s:3:"alt";s:0:"";}', 0),
(1122, 1114, 1367, 1, 'a:2:{s:5:"title";s:64:"Figure 2: Zeihl Neelsen stain was positive for acid fast bacilli";s:3:"alt";s:0:"";}', 1),
(1124, 1116, 1369, 1, 'a:2:{s:5:"title";s:114:"Figure 1: Diagram of face showing the location of the forehead swelling (abscess) with purulent discharge punctum.";s:3:"alt";s:0:"";}', 0),
(1124, 1116, 1370, 1, 'a:2:{s:5:"title";s:132:"Figure 2: MRI Brain. Sagital view. Evidence of retro-bulbar collection at right orbit (Arrow: collection at right retro-bulbar area)";s:3:"alt";s:0:"";}', 1),
(1129, 1121, 1372, 1, 'a:2:{s:5:"title";s:65:"Figure 1 : Full blood picture showed marked eosinophilia (arrows)";s:3:"alt";s:0:"";}', 0),
(1129, 1121, 1374, 1, 'a:2:{s:5:"title";s:134:"Figure 2: Histopathological examination of the esophagus and gastric mucosae showed eosinophilic infiltration >20 per high power field";s:3:"alt";s:0:"";}', 1),
(1129, 1121, 1376, 1, 'a:2:{s:5:"title";s:134:"Figure 2: Histopathological examination of the esophagus and gastric mucosae showed eosinophilic infiltration >20 per high power field";s:3:"alt";s:0:"";}', 2),
(1129, 1121, 1378, 1, 'a:2:{s:5:"title";s:145:"Figure 3: Colonoscopy showed mild ileitis with pancolitis, predominantly involving the descending, sigmoid colon with no loss of vascular pattern";s:3:"alt";s:0:"";}', 3),
(1129, 1121, 1380, 1, 'a:2:{s:5:"title";s:145:"Figure 3: Colonoscopy showed mild ileitis with pancolitis, predominantly involving the descending, sigmoid colon with no loss of vascular pattern";s:3:"alt";s:0:"";}', 4),
(1129, 1121, 1382, 1, 'a:2:{s:5:"title";s:179:"Figure 4: Histopathological examination of rectum and terminal ileum showed presence of mild to moderate eosinophilic infiltration of thelamina propia of > 30 per high power field";s:3:"alt";s:0:"";}', 5),
(1129, 1121, 1383, 1, 'a:2:{s:5:"title";s:179:"Figure 4: Histopathological examination of rectum and terminal ileum showed presence of mild to moderate eosinophilic infiltration of thelamina propia of > 30 per high power field";s:3:"alt";s:0:"";}', 6),
(1133, 1125, 1386, 1, 'a:2:{s:5:"title";s:274:"Figure 1: Photograph showing variant origin of common hepatic and middle colic artery. CT-coeliac trunk , L-left gastric artery, S-splenic artery, CHA-Common hepatic artery, ST-Stomach, MCA-Middle colic artery, A-Aorta, IVC-Inferior vena cava, SMA-Superior mesenteric artery";s:3:"alt";s:0:"";}', 0),
(1133, 1125, 1387, 1, 'a:2:{s:5:"title";s:338:"Figure 2: Photograph showing the middle colic artery originating from splenic artery and passing posterior to the body of pancreas to reach transverse colon. S-Splenic artery, MCA-Middle colic artery, P-Pancreas, TC-Transverse colon , ST-Stomach , LV-Liver, L-Left gastric artery, CHA-Common hepatic artery, SMA-Superior mesenteric artery";s:3:"alt";s:0:"";}', 1),
(1166, 1158, NULL, NULL, NULL, 0),
(1172, 1164, NULL, NULL, NULL, 0),
(1177, 1169, NULL, NULL, NULL, 0),
(1182, 1174, 1482, 1, 'a:2:{s:5:"title";s:59:"Figure 1: National Stroke Association Stroke Risk Scorecard";s:3:"alt";s:0:"";}', 0),
(1182, 1174, 1483, 1, 'a:2:{s:5:"title";s:60:"Figure 2: National Stroke Association Risk Scorecard Results";s:3:"alt";s:0:"";}', 1),
(1182, 1174, 1484, 1, 'a:2:{s:5:"title";s:63:"Figure 3: Knowledge of respodents of the risk factors of stroke";s:3:"alt";s:0:"";}', 2),
(1182, 1174, 1485, 1, 'a:2:{s:5:"title";s:31:"Figure 4: Source of information";s:3:"alt";s:0:"";}', 3),
(1194, 1186, NULL, NULL, NULL, 0),
(1200, 1192, NULL, NULL, NULL, 0),
(1202, 1194, NULL, NULL, NULL, 0),
(1205, 1197, 1474, 1, 'a:2:{s:5:"title";s:43:"Figure 1: Images of siblings affected by CP";s:3:"alt";s:0:"";}', 0),
(1205, 1197, 1475, 1, 'a:2:{s:5:"title";s:25:"Figure 2: Family genogram";s:3:"alt";s:0:"";}', 1),
(1211, 1203, NULL, NULL, NULL, 0),
(1215, 1207, 1477, 1, 'a:2:{s:5:"title";s:131:"Figure 1: Area of swelling and necrotic patch noted over anterior abdominal wall extending downwards from the chest wall superiorly";s:3:"alt";s:0:"";}', 0),
(1215, 1207, 1478, 1, 'a:2:{s:5:"title";s:161:"Figure 2: Another picture depicting the area of swelling and necrotic patch noted over anterior abdominal wall extending downwards from the chest wall superiorly";s:3:"alt";s:0:"";}', 1),
(1215, 1207, 1479, 1, 'a:2:{s:5:"title";s:149:"Figure 3: Coronal view of a CT scan. Blue arrow demonstrating the abscess collection tracking downwards from the chest wall to the left lumbar region";s:3:"alt";s:0:"";}', 2),
(1215, 1207, 1480, 1, 'a:2:{s:5:"title";s:119:"Figure 4: Axial CT scan view. Blue arrow showing the ruptured left silicone implant with surrounding abscess collection";s:3:"alt";s:0:"";}', 3),
(1227, 1219, NULL, NULL, NULL, 0),
(1228, 1220, NULL, NULL, NULL, 0),
(1231, 1223, 1504, 1, 'a:2:{s:5:"title";s:162:"Figure 1: Photographs showing the measurement of the parameters (AB-intercondylar notch depth; CD-intercondylar notch width; EF-condylar width; GH-condylar depth)";s:3:"alt";s:0:"";}', 0),
(1231, 1223, 1505, 1, 'a:2:{s:5:"title";s:110:"Figure 2: Photographs showing 2A-inverted ‘U’ shaped and 2B-inverted ‘V’ shaped intercondylar notches.";s:3:"alt";s:0:"";}', 1),
(1234, 1226, NULL, NULL, NULL, 0),
(1241, 1233, NULL, NULL, NULL, 0),
(1248, 1240, NULL, NULL, NULL, 0),
(1252, 1244, NULL, NULL, NULL, 0),
(1255, 1247, NULL, NULL, NULL, 0),
(1258, 1250, NULL, NULL, NULL, 0),
(1264, 1256, 1513, 1, 'a:2:{s:5:"title";s:146:"Figure 1a: Contrast enhanced CT angiogram of the thorax reveals an intimal flap within the descending aorta giving rise to a true and false lumen.";s:3:"alt";s:0:"";}', 0),
(1264, 1256, 1514, 1, 'a:2:{s:5:"title";s:77:"Figure 1b: Chest radiograph day 2 post TEVAR with endovascular stent in situ.";s:3:"alt";s:0:"";}', 1),
(1267, 1259, NULL, NULL, NULL, 0),
(1275, 1267, 1523, 1, 'a:2:{s:5:"title";s:376:"Figure 1: MRI of the brain in sagital T2WI and contrast-enhanced T1 MPRAGE demonstrate multiple serpiginous flow voids (star and bent arrow) representing dilated venous channels. Abnormal vessels (bent arrow) are seen draining into a dilated vein (thin arrow), which in turn drains into the superior sagital sinus. Narrowing of aqueduct of sylvius is marked with an arrowhead.";s:3:"alt";s:0:"";}', 0),
(1316, 1307, NULL, NULL, NULL, 0),
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(6045, 6036, NULL, NULL, NULL, 0),
(6052, 6043, 1698, 1, 'a:2:{s:5:"title";s:344:"Figure 1: SDS-PAGE and immunoblotting profiles of raw (a) and cooked (b) extracts of mud crab. Lane M indicates molecular weight markers in kiloDalton (kDa). Lane B and N represent a blank and a negative control of a normal serum, respectively. Lane 1 through 21 represents immunoblotting results of sera from 21 patients with mud crab allergy.";s:3:"alt";s:0:"";}', 0),
(6052, 6043, 1699, 1, 'a:2:{s:5:"title";s:344:"Figure 1: SDS-PAGE and immunoblotting profiles of raw (a) and cooked (b) extracts of mud crab. Lane M indicates molecular weight markers in kiloDalton (kDa). Lane B and N represent a blank and a negative control of a normal serum, respectively. Lane 1 through 21 represents immunoblotting results of sera from 21 patients with mud crab allergy.";s:3:"alt";s:0:"";}', 1),
(6052, 6043, 1701, 1, 'a:2:{s:5:"title";s:132:"Table 1 : Immunoblotting results of raw (a) and cooked (b) extracts of mud crab using sera from 21 patients with allergy to mud crab";s:3:"alt";s:0:"";}', 2),
(6052, 6043, 1702, 1, 'a:2:{s:5:"title";s:132:"Table 1 : Immunoblotting results of raw (a) and cooked (b) extracts of mud crab using sera from 21 patients with allergy to mud crab";s:3:"alt";s:0:"";}', 3),
(6053, 6044, NULL, NULL, NULL, 0),
(6057, 6048, 1706, 1, 'a:2:{s:5:"title";s:38:"Figure 1: Ultrasoud image of fracture ";s:3:"alt";s:0:"";}', 0),
(6057, 6048, 1707, 1, 'a:2:{s:5:"title";s:35:"Figure 2: X-ray image of a fracture";s:3:"alt";s:0:"";}', 1),
(6057, 6048, 1708, 1, 'a:2:{s:5:"title";s:68:"Figure 3: Data collection over the designated data collection period";s:3:"alt";s:0:"";}', 2),
(6059, 6050, 1710, 1, 'a:2:{s:5:"title";s:130:"Figure 1: A representative histogram plot of an estimated 1% artificial mixture showing a population of fetal cells in adult cells";s:3:"alt";s:0:"";}', 0),
(6059, 6050, 1711, 1, 'a:2:{s:5:"title";s:156:"Figure 2: Linearity study showing the correlation graph between the FMH estimated by the flow cytometry (y-axis) and the theoretically estmated FMH (x-axis)";s:3:"alt";s:0:"";}', 1),
(6059, 6050, 1712, 1, 'a:2:{s:5:"title";s:160:"Figure 3: Linearity study showing the correlation graph between the FMH estimated by the Kleihauer’s test (y-axis) and the theoretically estmated FMH (x-axis)";s:3:"alt";s:0:"";}', 2),
(6059, 6050, 1714, 1, 'a:2:{s:5:"title";s:149:"Figure 4: Linearity study showing the correlation graph between the FMH estimated by the flow cytometry (x-axis) and the Kleihauer’s test (y-axis).";s:3:"alt";s:0:"";}', 3),
(6059, 6050, 1715, 1, 'a:2:{s:5:"title";s:126:"Figure 5: The correlation graph between the % fetal cells estimated by flow cytometry (x-axis) and the Kleihauer test (y-axis)";s:3:"alt";s:0:"";}', 4),
(6059, 6050, 1716, 1, 'a:2:{s:5:"title";s:112:"Figure 6: The correlation graph between the estimated FMH by flow cytometry (x-axis) and Kleihauer test (y-axis)";s:3:"alt";s:0:"";}', 5),
(6061, 6052, 1717, 1, 'a:2:{s:5:"title";s:109:"Figure 1: A sagittal view of preoperative CT scan showing a tumour occupying the nasopharyngeal space (arrow)";s:3:"alt";s:0:"";}', 0),
(6061, 6052, 1719, 1, 'a:2:{s:5:"title";s:150:"Figure 2 : A coronal view of CT scan showing an intact skull base with no evidence of bony erosion or destruction by the nasopharyngeal tumour (arrow)";s:3:"alt";s:0:"";}', 1),
(6062, 6053, 1722, 1, 'a:2:{s:5:"title";s:245:"Figure 1: A diagram illustrating the various approaches to the homicide-suicide. Adapted from “Homicide followed by suicide: an empirical analysis” by MCA Liem, 2010, Dissertation Utrecht University, the Netherlands. Adapted with permission.";s:3:"alt";s:0:"";}', 0),
(6066, 6057, NULL, NULL, NULL, 0),
(6073, 6064, 1732, 1, 'a:2:{s:5:"title";s:161:"Figure 1: Analysis of PfNT3 PCR products on 0.8% agarose gel electrophoresis. Lane 1-Molecular marker lane; Lane 2 & 3-1.3Kb PCR product; Lane 4-negative control";s:3:"alt";s:0:"";}', 0),
(6073, 6064, 1733, 1, 'a:2:{s:5:"title";s:184:"Figure 2: Analysis of PfNT3-1-108 sequence PCR products on 1.5% agarose gel electrophoresis. Lane 1-Molecular marker lane; Lane 2 - 5-PCR product of N-terminal; Lane 6-negative control";s:3:"alt";s:0:"";}', 1),
(6073, 6064, 1734, 1, 'a:2:{s:5:"title";s:132:"Figure 3: Homology study by alignment of PF14_0662 amino acid sequence with other nucleoside transporter homologues (By ClustalW2.0)";s:3:"alt";s:0:"";}', 2),
(6073, 6064, 1735, 1, 'a:2:{s:5:"title";s:179:"Figure 4: Phylogenetic tree showing relationship between PF14_0662 with other nucleoside transporter proteins by Phylodraw ver. 0.8. PF14_0662-P. falciparum nucleoside transporter";s:3:"alt";s:0:"";}', 3),
(6073, 6064, 1736, 1, 'a:2:{s:5:"title";s:80:"Figure 5: Topology prediction of PfNT3 by using TMHMM, an ExPASy Proteomics tool";s:3:"alt";s:0:"";}', 4),
(6073, 6064, 1737, 1, 'a:2:{s:5:"title";s:124:"Figure 6: Analysis of expressed PfNT3 recombinant protein on 10% SDS PAGE. Lane 1-Expressed protein; Lane 2-Purified protein";s:3:"alt";s:0:"";}', 5),
(6073, 6064, 1738, 1, 'a:2:{s:5:"title";s:250:"Figure 7: Specificity of PfNT3 antibodies. Immunoblot analysis with antibodies against A-Purified fusion protein (antigen); B-NH2-terminal sequence after thrombin digestion; C-Lane 1-GST (negative control), Lane 2-Membrane fraction; D-Preimmune serum";s:3:"alt";s:0:"";}', 6),
(6073, 6064, 1739, 1, 'a:2:{s:5:"title";s:123:"Figure 8: Smear analysis by deconvoluted immunofluorescence microscopy of PfNT3 in P. falciparum infected red blood cells. ";s:3:"alt";s:0:"";}', 7),
(6073, 6064, 1740, 1, 'a:2:{s:5:"title";s:310:"Figure 9: Transmission Electron Micrograph of ultrathin cryo-sections of the intraerythrocytic trophozoite stage of P. falciparum using PfNT3 antibodies. Magnification is x12000 Immunogold labelling (10-nm gold particles) of PfNT3 antibodies bound to intraerythrocyte P. falciparum is depicted with arrowheads.";s:3:"alt";s:0:"";}', 8),
(6078, 6069, NULL, NULL, NULL, 0),
(6081, 6072, 1743, 1, 'a:2:{s:5:"title";s:255:"Figure 1: Intraoperative photograph of dissected giant appendicular abscess with brown faecolith and not the usual straw colored fluid content mimicking a caecal tumour and ceacal diverticulitis on clinical presentation and needle aspiration respectively.";s:3:"alt";s:0:"";}', 0),
(6087, 6078, 1746, 1, 'a:2:{s:5:"title";s:31:"Figure 1: Giant bariatric stent";s:3:"alt";s:0:"";}', 0),
(6087, 6078, 1748, 1, 'a:2:{s:5:"title";s:42:"Figure 2: Fundal lesion with small bubbles";s:3:"alt";s:0:"";}', 1),
(6087, 6078, 1749, 1, 'a:2:{s:5:"title";s:57:"Figure 3: UGI contrast study showing leak at angle of his";s:3:"alt";s:0:"";}', 2),
(6087, 6078, 1751, 1, 'a:2:{s:5:"title";s:62:"Figure 4: Peri-splenic collection (CT scan with oral contrast)";s:3:"alt";s:0:"";}', 3),
(7257, 7248, NULL, NULL, NULL, 0),
(7261, 7252, 1893, 1, 'a:2:{s:5:"title";s:51:"Figure 1: Mean of family members needs met or unmet";s:3:"alt";s:0:"";}', 0),
(7268, 7259, NULL, NULL, NULL, 0),
(7270, 7261, NULL, NULL, NULL, 0),
(7273, 7264, NULL, NULL, NULL, 0),
(7280, 7271, NULL, NULL, NULL, 0),
(7283, 7274, NULL, NULL, NULL, 0),
(7291, 7282, 1894, 1, 'a:2:{s:5:"title";s:39:"Figure 1: Percentage of cell phenotypes";s:3:"alt";s:0:"";}', 0),
(7295, 7286, NULL, NULL, NULL, 0),
(7297, 7288, NULL, NULL, NULL, 0),
(7300, 7291, NULL, NULL, NULL, 0),
(7303, 7294, 1908, 1, 'a:2:{s:5:"title";s:36:"Figure 1: Family tree of the patient";s:3:"alt";s:0:"";}', 0),
(7306, 7297, 1909, 1, 'a:2:{s:5:"title";s:123:"Figure 1: Endoscopic photograph of the larynx showing a subglottic cyst at left posterior commissure with subglottic oedema";s:3:"alt";s:0:"";}', 0),
(7309, 7300, 1910, 1, 'a:2:{s:5:"title";s:127:"Figure 1: Right hand showing a well-defined erythematous patch with vesicles of varying sizes on the medial edge of the lesion.";s:3:"alt";s:0:"";}', 0),
(7309, 7300, 1911, 1, 'a:2:{s:5:"title";s:164:"Figure 2: Photograph showing right hand after 3 weeks of treatment. A small area of post-inflamatory hyperpigmentation WAS noted at the site of the previous lesion.";s:3:"alt";s:0:"";}', 1),
(7313, 7304, 1912, 1, 'a:2:{s:5:"title";s:141:"Figure 1: A 3 l saline irrigation bag with stopper is placed in one tube. A piece of suction tubing connects the other end to a 3-way stopper";s:3:"alt";s:0:"";}', 0),
(7313, 7304, 1913, 1, 'a:2:{s:5:"title";s:202:"Figure 2: A second piece of suction tubing connects the 3-way stopper to a sphygmomanometer bulb for intra-operative inflation and deflation. Completed inflatable air bag for patient positioning (below)";s:3:"alt";s:0:"";}', 1),
(67602, 67588, 2825, 1, 'a:2:{s:5:"title";s:148:"Figure 1: a) whole plant of Gelam tree b) Gelam ‘papery’ and flaky bark c) leaves of Gelam tree d) brown capsules of Gelam fruits (Burkill 1993)";s:3:"alt";s:0:"";}', 0),
(67605, 67591, 2826, 1, 'a:2:{s:5:"title";s:149:"Figure 1: a) Radiograph of fifth metatarsal fracture (Tan et al. 2016). b) Diagram of fifth metatarsal showing related tendons and fractures’ zones";s:3:"alt";s:0:"";}', 0),
(67605, 67591, 2827, 1, 'a:2:{s:5:"title";s:93:"Figure 2: Point reduction clamp is used to prevent displacement and gapping (Tan et al. 2016)";s:3:"alt";s:0:"";}', 1),
(67610, 67596, NULL, NULL, NULL, 0),
(67614, 67600, NULL, NULL, NULL, 0),
(67618, 67604, 2828, 1, 'a:2:{s:5:"title";s:134:"Figure 1A: Effect of white rice, brown rice, black rice, sweet corn and baby corn extract with water on cell viability in HT-22 cells.";s:3:"alt";s:0:"";}', 0),
(67618, 67604, 2830, 1, 'a:2:{s:5:"title";s:136:"Figure 1B: Effect of white rice, brown rice, black rice, sweet corn and baby corn extract with ethanol on cell viability in HT-22 cells.";s:3:"alt";s:0:"";}', 1),
(67618, 67604, 2831, 1, 'a:2:{s:5:"title";s:137:"Figure 1C :  Effect of white rice, brown rice, black rice, sweet corn and baby corn extract with hexane on cell viability in HT-22 cells.";s:3:"alt";s:0:"";}', 2),
(67618, 67604, 2832, 1, 'a:2:{s:5:"title";s:120:"Figure 2: Exposure of HT-22 cells to H2O2  over concentration range 31-2000 µM for 24 h was evaluated using MTT assay. ";s:3:"alt";s:0:"";}', 3),
(67618, 67604, 2833, 1, 'a:2:{s:5:"title";s:268:"Figure 3A:  Protective effect of white rice, brown rice, black rice, sweet corn and baby corn extract with water against 250 µM H2O2. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 4),
(67618, 67604, 2834, 1, 'a:2:{s:5:"title";s:270:"Figure 3B:  Protective effect of white rice, brown rice, black rice, Sweet corn and Baby corn extract with ethanol against 250 µM H2O2. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 5),
(67618, 67604, 2835, 1, 'a:2:{s:5:"title";s:267:"Figure 3C: Protective effect of white rice, brown rice, black rice, sweet corn and baby corn extract with hexane gainst 250 μM H2O2. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 6),
(67618, 67604, 2836, 1, 'a:2:{s:5:"title";s:505:"Figure 4A: Flow cytometric analysis type of cell death in H2O2-treated HT-22 cells. Cell, untreated HT-22 cells; 250 μM H2O2,HT-22 cells treated  with 250 μM H2O2 for 24 h; QC, exposure of HT-22 cells to 250 μM H2O2 over 24 h in the 24h pre-treated 10 μM QC, 100 μg/mL BrE, BrHe, BlE, BlHe, BaH, BaE, BaHe, SH, SE and SHe. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01) (BrE; Brown rice extract with ethanol, BrHe";s:3:"alt";s:0:"";}', 7),
(67618, 67604, 2838, 1, 'a:2:{s:5:"title";s:364:"Figure 4B : Flow cytometric analysis type of cell death in H2O2-treated HT-22 cells. Cell, untreated HT-22 cells; 250 µM H2O2,HT-22 cells treated  with 250 µM H2O2 for 24 h; QC, exposure of HT-22 cells to 250 µM H2O2 over 24 h in the 24h pre-treated 10 µM QC, 100 µg/mL BrE, BrHe, BlE, BlHe, BaH, BaE, BaHe, SH, SE and SHe.  (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 8),
(67618, 67604, 2839, 1, 'a:2:{s:5:"title";s:500:"Figure 5A: (a) : Cell control, (b) : Cell + 250 uM H2O2 , (c) : Cell + 10 uM Quercitin + 250uM H2O2,   (d) : Cell + 100 ug/mL Black rice/EtOH + 250uM H2O2, (e) : Cell + 100 ug/mL Baby corn/H2O + 250uM H2O2, (f) : Cell + 100 ug/mL Baby corn/EtOH + 250uM H2O2. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01) (BlE; Black rice extract with ethanol, BaH; Baby corn extract with water, BaE; Baby corn extract with ethan";s:3:"alt";s:0:"";}', 9),
(67618, 67604, 2840, 1, 'a:2:{s:5:"title";s:502:"Figure 5B : The protective effect of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) on H2O2 induced production of ROS in HT-22 cells. The cells were pretreated with 100 μg/mL of black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) for 24 h before being treated with 250 μM H2O2 together with DCFDA for 30 min. The significance is between the indicated group and the group treated with hydroge";s:3:"alt";s:0:"";}', 10),
(67618, 67604, 2841, 1, 'a:2:{s:5:"title";s:455:"Figure 6 : Real-time PCR for quantification of BDNF expression levels following exposure to 250 μM H2O2, 100 μg/mL black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) + 250 μM H2O2. The fold change was calculated based on normalization with β-actin gene expression. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 11),
(67618, 67604, 2842, 1, 'a:2:{s:5:"title";s:454:"Figure 7: Western blot analysis of BDNF protein expression levels following exposure to 250 μM H2O2, 100 μg/mL black glutinous rice extracts with ethanol (BlE), baby corn extracts with water (BaH) and ethanol (BaE) + 250 μM H2O2. The fold change was calculated based on normalization with β-actin protein expression. The significance is between the indicated group and the group treated with hydrogen peroxide only. (* = p < 0.05) and (** = p < 0.01)";s:3:"alt";s:0:"";}', 12),
(67628, 67614, 2843, 1, 'a:2:{s:5:"title";s:103:"Figure 1: Measurement of systolic blood pressure in normal and cholesterol diet fed ovariectomised rats";s:3:"alt";s:0:"";}', 0),
(67628, 67614, 2844, 1, 'a:2:{s:5:"title";s:104:"Figure 2: Measurement of diastolic blood pressure in normal and cholesterol diet fed ovariectomised rats";s:3:"alt";s:0:"";}', 1),
(67628, 67614, 2845, 1, 'a:2:{s:5:"title";s:99:"Figure 3: Measurement of mean blood pressure in normal and cholesterol diet fed ovariectomised rats";s:3:"alt";s:0:"";}', 2),
(67631, 67617, NULL, NULL, NULL, 0),
(67654, 67640, 2846, 1, 'a:2:{s:5:"title";s:272:"Figure 1: Positive (IHC) detection of p53 protein (A-D). Tissue with crisp brownish nuclear precipitatation of p53 protein overexpression; (A) S0045164, magnification x40, (B) S004776, magnification x10. (C) S0048960, magnification x10, and (D) S0047962, magnification x40";s:3:"alt";s:0:"";}', 0),
(67654, 67640, 2848, 1, 'a:2:{s:5:"title";s:272:"Figure 1: Positive (IHC) detection of p53 protein (A-D). Tissue with crisp brownish nuclear precipitatation of p53 protein overexpression; (A) S0045164, magnification x40, (B) S004776, magnification x10. (C) S0048960, magnification x10, and (D) S0047962, magnification x40";s:3:"alt";s:0:"";}', 1),
(67654, 67640, 2849, 1, 'a:2:{s:5:"title";s:272:"Figure 1: Positive (IHC) detection of p53 protein (A-D). Tissue with crisp brownish nuclear precipitatation of p53 protein overexpression; (A) S0045164, magnification x40, (B) S004776, magnification x10. (C) S0048960, magnification x10, and (D) S0047962, magnification x40";s:3:"alt";s:0:"";}', 2),
(67654, 67640, 2850, 1, 'a:2:{s:5:"title";s:272:"Figure 1: Positive (IHC) detection of p53 protein (A-D). Tissue with crisp brownish nuclear precipitatation of p53 protein overexpression; (A) S0045164, magnification x40, (B) S004776, magnification x10. (C) S0048960, magnification x10, and (D) S0047962, magnification x40";s:3:"alt";s:0:"";}', 3),
(67662, 67648, 2851, 1, 'a:2:{s:5:"title";s:110:"Figure 1: Peripapillary retinal nerve fiber layer thickness changes before and after laser in the argon group.";s:3:"alt";s:0:"";}', 0),
(67662, 67648, 2852, 1, 'a:2:{s:5:"title";s:111:"Figure 2: Peripapillary retinal nerve fiber layer thickness changes before and after laser in the PASCAL group.";s:3:"alt";s:0:"";}', 1),
(67667, 67653, 2853, 1, 'a:2:{s:5:"title";s:70:"Figure 1: Mean serum MMP-3 levels in the various groups of RA patients";s:3:"alt";s:0:"";}', 0),
(67674, 67660, 2854, 1, 'a:2:{s:5:"title";s:92:"Figure 1: Single PPGF Plot (Blue: 19 year old healthy reference pulse; Red: target subject).";s:3:"alt";s:0:"";}', 0),
(67674, 67660, 2855, 1, 'a:2:{s:5:"title";s:167:"Figure 2: Aortic blood pressure waveform (AP=augmentation pressure, PP=pulse pressure, SBP=systolic blood pressure, DBP=diastolic blood pressure, P1=forward pressure).";s:3:"alt";s:0:"";}', 1),
(67680, 67666, 2856, 1, 'a:2:{s:5:"title";s:421:"Figure 1: A) Photomicrograph of hippocampus CA1 stained with H&E (x100). B) Photomicrograph in higher magnification (x200). PB2 shows shrunken apoptotic neurons (darkly stained). PB2T shows reduced number of atypically morphology cells compared to PB2, but disorganized arrangement of cells compared to CTRL. Str. or.: stratum oriens, Str. rad.: stratum radiatum, Str. l-m: stratum lacunosum molecular, DG: dentate gyrus.";s:3:"alt";s:0:"";}', 0),
(67680, 67666, 2857, 1, 'a:2:{s:5:"title";s:340:"Figure 2: A) Photomicrograph of hippocampus CA3 stained with H&E (x100). B) Photomicrograph in higher magnification (x400). Normal neuronal cells were observed in CTRL. B (i). PB2. B (ii) shows apoptotic neurons while PB2T. B (iii) shows reduced number of atypical cells. Disorganization of neuronal cells markedly observed in PB2 and PB2T.";s:3:"alt";s:0:"";}', 1),
(67680, 67666, 2858, 1, 'a:2:{s:5:"title";s:135:"Figure 3: Bar graph of brain lead level (μg/g brain). *PB2 shows significant difference of brain lead levels compared to PB2 and CTRL.";s:3:"alt";s:0:"";}', 2),
(67680, 67666, 2859, 1, 'a:2:{s:5:"title";s:134:"Figure 4: Bar graph of brain TRF level (μg/ml brain). *PB2 shows significant difference of brain TRF levels compared to PB2 and CTRL.";s:3:"alt";s:0:"";}', 3),
(67680, 67666, 2860, 1, 'a:2:{s:5:"title";s:154:"Figure 5: Bar graph of erythrocyte SOD activity (U/mghemoglobin). *PB2T shows significant difference of erythrocyte SOD activity compared to PB2 and CTRL.";s:3:"alt";s:0:"";}', 4),
(67680, 67666, 2861, 1, 'a:2:{s:5:"title";s:117:"Figure 6: Bar graph of brain MDA level (μM). *PB2 shows significant difference of brain MDA levels compared to CTRL.";s:3:"alt";s:0:"";}', 5),
(67688, 67674, 2862, 1, 'a:2:{s:5:"title";s:49:"Figure 1: Location of polypoidal lesions on ICG14";s:3:"alt";s:0:"";}', 0),
(67688, 67674, 2863, 1, 'a:2:{s:5:"title";s:91:"Figure 2: Polyp(s) configuration, (a) Diploid, (b) Clusters, and (c) ‘String of pearls’";s:3:"alt";s:0:"";}', 1),
(67688, 67674, 2864, 1, 'a:2:{s:5:"title";s:161:"Figure 3: Fundus photograph of a PCV patient showing multiple drusens (black arrows), with hemorrhagic PED (white arrow) and subretinal hemorrhages (red arrows).";s:3:"alt";s:0:"";}', 2),
(67688, 67674, 2865, 1, 'a:2:{s:5:"title";s:146:"Figure 4: SD-OCT showing double-layer sign (black arrow), exudative changes (white arrows), double-hump sign (red arrow) and PEDs (yellow arrows).";s:3:"alt";s:0:"";}', 3),
(67691, 67677, 2866, 1, 'a:2:{s:5:"title";s:92:"Figure 1: Number of CAM products or practices used by the CAM-DM group in the past 12 months";s:3:"alt";s:0:"";}', 0),
(67691, 67677, 2867, 1, 'a:2:{s:5:"title";s:91:"Figure 2: Number of CAM products or practices used by the CAM-G group in the past 12 months";s:3:"alt";s:0:"";}', 1),
(67693, 67679, 2868, 1, 'a:2:{s:5:"title";s:100:"Figure 1: A sample of healed tibia extracted from the rat with T plate in place to fix the fracture.";s:3:"alt";s:0:"";}', 0),
(67693, 67679, 2871, 1, 'a:2:{s:5:"title";s:175:"Figure 2: An area which consisted of 100 axial slices above and below fracture line were chosen as the region of interest (ROI) to be scanned for fracture healing assessments.";s:3:"alt";s:0:"";}', 1),
(67693, 67679, 2870, 1, 'a:2:{s:5:"title";s:248:"Figure 3: Three dimensions (3D) images of the bridging cortices generated by micro-CT. SHAM: sham-operated, ORX: orchidectomised-control, TEN: orchidectomy + testosterone enanthate treatment 7 mg/rat, EL: orchidectomy + Eurycoma longifolia 15 mg/kg";s:3:"alt";s:0:"";}', 2),
(67693, 67679, 2872, 1, 'a:2:{s:5:"title";s:384:"Figure 4: Fraction of mineralized tissue of the bridging cortices (BVbc/TVbc) after 6 weeks of study. Results were expressed as mean ± standard error mean (SEM). Similar alphabets showed significant difference. SHAM: Sham-operated, ORX: orchidectomised-control, TEN: orchidectomy + testosterone enanthate treatment 7 mg/rat, EL: orchidectomy + Eurycoma longifolia treatment 15 mg/kg.";s:3:"alt";s:0:"";}', 3),
(67693, 67679, 2873, 1, 'a:2:{s:5:"title";s:295:"Figure 5: The mBMDbc between the groups after 6 weeks of study. Results were expressed as mean ± standard error mean (SEM). SHAM: Sham-operated, ORX: orchidectomised-control, TEN: orchidectomy + testosterone enanthate treatment 7mg/rat, EL: orchidectomy + Eurycoma longifolia treatment 15mg/kg.";s:3:"alt";s:0:"";}', 4),
(67693, 67679, 2874, 1, 'a:2:{s:5:"title";s:309:"Figure 6: The soft callus volume values of the groups after 6 weeks of study. Results were expressed as mean ± standard error mean (SEM). SHAM: Sham-operated, ORX: orchidectomised-control, TEN: orchidectomy + testosterone enanthate treatment 7mg/rat, EL: orchidectomy + Eurycoma longifolia treatment 15mg/kg.";s:3:"alt";s:0:"";}', 5),
(67696, 67682, 2875, 1, 'a:2:{s:5:"title";s:63:"Figure 1: General protocol for bone histomorphometric analysis.";s:3:"alt";s:0:"";}', 0),
(67696, 67682, 2876, 1, 'a:2:{s:5:"title";s:48:"Figure 2: Processing of decalcified bone samples";s:3:"alt";s:0:"";}', 1),
(67696, 67682, 2877, 1, 'a:2:{s:5:"title";s:50:"Figure 3: Haematoxylin and eosin staining protocol";s:3:"alt";s:0:"";}', 2),
(67696, 67682, 2878, 1, 'a:2:{s:5:"title";s:50:"Figure 4: Processing of undecalcified bone samples";s:3:"alt";s:0:"";}', 3),
(67696, 67682, 2879, 1, 'a:2:{s:5:"title";s:89:"Figure 5: The Weibel grid composed of line system (21 lines) and point system (42 points)";s:3:"alt";s:0:"";}', 4),
(67696, 67682, 2880, 1, 'a:2:{s:5:"title";s:231:"Figure 6: Micrographic photos of decalcified trabecular bone stained with haematoxylin and eosin (20x magnification) demonstrating cellular bone histomorphomertic parameters. Oc, osteoclast; Ob, osteoblast; and ES, eroded surfaces.";s:3:"alt";s:0:"";}', 5),
(67696, 67682, 2881, 1, 'a:2:{s:5:"title";s:500:"Figure 7:	Sample of trabecular section stained with haematoxylin and eosin (20x magnification superimposed with multipurpose test-system (M42 grid). LTb, area of interaction between trabeculae and the test line; PTb, area of interaction between trabeculae and the test point; LOb, area of interaction between osteoblast cell and test line; LOc, area ofinteraction between osteoclast cell and test line; LES, area of interaction between eroded surface and test line; LO, area of interaction between os";s:3:"alt";s:0:"";}', 6),
(67696, 67682, 2882, 1, 'a:2:{s:5:"title";s:243:"Figure 8: Micrographic photo of decalcified trabecular labelled with calcein (20x magnification) demonstrating dynamic bone histomorphometric parameters. dLS, double-labelled surface; sLS, single-labelled surface; MAR, mineral apposition rate.";s:3:"alt";s:0:"";}', 7),
(67696, 67682, 2883, 1, 'a:2:{s:5:"title";s:276:"Figure 9: Sample of trabecular section labelled with calcein (20x magnification superimposed with multipurpose test-system (M42 grid). LsLS, area of interaction between single-labelled surface and test line; LdLS, area of interaction between double-labelled surface and test. ";s:3:"alt";s:0:"";}', 8),
(67701, 67687, 2884, 1, 'a:2:{s:5:"title";s:158:"Figure 1: Color fundus photographs of right and left eyes. Right fundus showed multiple deep hypopigmented lesions with a dull macula. Left fundus was normal.";s:3:"alt";s:0:"";}', 0),
(67701, 67687, 2885, 1, 'a:2:{s:5:"title";s:186:"Figure 2: SD-OCT of right and left eyes. OCT of right eye showed generalised thinning, disruption of photoreceptors with cavitations and RPE interdigitations. OCT of left eye was normal.";s:3:"alt";s:0:"";}', 1),
(67701, 67687, 2886, 1, 'a:2:{s:5:"title";s:134:"Figure 3: IR of right and left eyes. IR of right eye showed multiple hyper-reflective areas at the macula region. Left eye was normal.";s:3:"alt";s:0:"";}', 2),
(67701, 67687, 2887, 1, 'a:2:{s:5:"title";s:182:"Figure 4: FAF of right and left eyes. FAF of right eye showed a rim of hypoautofluorescence surrounding the fovea with intervening area of hyperautofluorescence. Left eye was normal.";s:3:"alt";s:0:"";}', 3),
(67704, 67690, 2888, 1, 'a:2:{s:5:"title";s:72:"Figure 1: Drawing of the amputated penile of the 31-year old Rohigya man";s:3:"alt";s:0:"";}', 0),
(67708, 67694, 2889, 1, 'a:2:{s:5:"title";s:140:"Figure 1: The figure shows a) the patient’s tracheostomy tube protruded anteriorly from stoma due to presence of hypertrophic scar (arrow)";s:3:"alt";s:0:"";}', 0),
(67708, 67694, 2890, 1, 'a:2:{s:5:"title";s:155:"Figure 1: The figure shows  b) close up view of the hypertrophic skin after the tracheostomy tube was changed to a microlaryngeal tube size 6 (white arrow)";s:3:"alt";s:0:"";}', 1),
(67708, 67694, 2891, 1, 'a:2:{s:5:"title";s:90:"Figure 1: The figure shows c) patient’s cuff tracheostomy tube that was dirty and mouldy";s:3:"alt";s:0:"";}', 2),
(67708, 67694, 2892, 1, 'a:2:{s:5:"title";s:150:"Figure 2: The figure shows a direct laryngoscopy view depicting distorted laryngeal structures with right arytenoid anteriorly displaced (white arrow)";s:3:"alt";s:0:"";}', 3),
(67715, 67701, 2893, 1, 'a:2:{s:5:"title";s:101:"Figure 1: Figure shows clinical apperance of CNPAS such as small chin, widened forehead, low set ears";s:3:"alt";s:0:"";}', 0),
(67715, 67701, 2894, 1, 'a:2:{s:5:"title";s:165:"Figure 2:  Figure shows an axial plane CT image at the level of the inferior meatus showing narrowing of the piriform aperture (width 5.4 mm) at the age of 1 months.";s:3:"alt";s:0:"";}', 1),
(67717, 67703, 2895, 1, 'a:2:{s:5:"title";s:97:"Figure 1: Anteroposterior radiograph of right shoulder: displaced fracture neck of right humerus.";s:3:"alt";s:0:"";}', 0),
(67717, 67703, 2896, 1, 'a:2:{s:5:"title";s:95:"Figure 2: Anteroposterior radiograph of left shoulder: displaced fracture neck of left humerus.";s:3:"alt";s:0:"";}', 1),
(67717, 67703, 2897, 1, 'a:2:{s:5:"title";s:119:"Figure 3: Anteroposterior radiograph of right shoulder showing fixation of right proximal humerus fracture dislocation.";s:3:"alt";s:0:"";}', 2),
(67717, 67703, 2898, 1, 'a:2:{s:5:"title";s:117:"Figure 4: Anteroposterior radiograph of left shoulder showing fixation of right proximal humerus fracture dislocation";s:3:"alt";s:0:"";}', 3),
(67723, 67709, 2899, 1, 'a:2:{s:5:"title";s:151:"Figure 1: Right fundus examination revealed hemorrhagic retinitis along the inferotemporal vessel and vitreous condensation along superotemporal vessel";s:3:"alt";s:0:"";}', 0),
(67723, 67709, 2900, 1, 'a:2:{s:5:"title";s:67:"Figure 2: Resolving retinal lesion at one month following treatment";s:3:"alt";s:0:"";}', 1),
(67723, 67709, 2901, 1, 'a:2:{s:5:"title";s:130:"Figure 3: Reactivation of right eye CMV retinitis (arrow) that was approaching the macula. Artifacts were marked with asterisk (*)";s:3:"alt";s:0:"";}', 2),
(67723, 67709, 2902, 1, 'a:2:{s:5:"title";s:92:"Figure 4: Resolution of recurrent retinitis following four weeks of intravitreal Ganciclovir";s:3:"alt";s:0:"";}', 3),
(67727, 67713, 2903, 1, 'a:2:{s:5:"title";s:127:"Figure 1: Left facial tumour protruding from the orbital cavity displacing the eyeball downwards (eyeball not seen in picture) ";s:3:"alt";s:0:"";}', 0);

-- --------------------------------------------------------

--
-- Table structure for table `content_field_op_author`
--

CREATE TABLE IF NOT EXISTS `content_field_op_author` (
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--
-- Dumping data for table `content_field_op_author`
--

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-- --------------------------------------------------------

--
-- Table structure for table `content_field_op_documentcloud_doc`
--

CREATE TABLE IF NOT EXISTS `content_field_op_documentcloud_doc` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_documentcloud_doc_doc_id` varchar(255) DEFAULT NULL,
  `field_op_documentcloud_doc_url` varchar(255) DEFAULT NULL,
  `field_op_documentcloud_doc_title` varchar(255) DEFAULT NULL,
  `field_op_documentcloud_doc_description` text,
  `field_op_documentcloud_doc_thumb` varchar(255) DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_op_documentcloud_doc`
--

INSERT INTO `content_field_op_documentcloud_doc` (`vid`, `nid`, `delta`, `field_op_documentcloud_doc_doc_id`, `field_op_documentcloud_doc_url`, `field_op_documentcloud_doc_title`, `field_op_documentcloud_doc_description`, `field_op_documentcloud_doc_thumb`) VALUES
(15, 15, 0, NULL, NULL, NULL, NULL, NULL),
(18, 15, 0, NULL, NULL, NULL, NULL, NULL),
(19, 15, 0, NULL, NULL, NULL, NULL, NULL),
(29, 26, 0, NULL, NULL, NULL, NULL, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_field_op_related_mmedia`
--

CREATE TABLE IF NOT EXISTS `content_field_op_related_mmedia` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_related_mmedia_nid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_op_related_mmedia_nid` (`field_op_related_mmedia_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_op_related_mmedia`
--

INSERT INTO `content_field_op_related_mmedia` (`vid`, `nid`, `delta`, `field_op_related_mmedia_nid`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `content_field_op_slideshow_images`
--

CREATE TABLE IF NOT EXISTS `content_field_op_slideshow_images` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_slideshow_images_nid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_op_slideshow_images_nid` (`field_op_slideshow_images_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_op_slideshow_images`
--

INSERT INTO `content_field_op_slideshow_images` (`vid`, `nid`, `delta`, `field_op_slideshow_images_nid`) VALUES
(36, 33, 0, 31),
(36, 33, 1, 32);

-- --------------------------------------------------------

--
-- Table structure for table `content_field_previewer`
--

CREATE TABLE IF NOT EXISTS `content_field_previewer` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_previewer_uid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_previewer_uid` (`field_previewer_uid`)
) ENGINE=MyISAM DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_previewer`
--

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(68586, 68571, 2, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_field_related_content`
--

CREATE TABLE IF NOT EXISTS `content_field_related_content` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_related_content_nid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_related_content_nid` (`field_related_content_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_related_content`
--

INSERT INTO `content_field_related_content` (`vid`, `nid`, `delta`, `field_related_content_nid`) VALUES
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(67727, 67713, 0, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_field_revmanuscript`
--

CREATE TABLE IF NOT EXISTS `content_field_revmanuscript` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_revmanuscript_fid` int(11) DEFAULT NULL,
  `field_revmanuscript_list` tinyint(4) DEFAULT NULL,
  `field_revmanuscript_data` text,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_revmanuscript`
--

INSERT INTO `content_field_revmanuscript` (`vid`, `nid`, `delta`, `field_revmanuscript_fid`, `field_revmanuscript_list`, `field_revmanuscript_data`) VALUES
(764, 761, 0, NULL, NULL, NULL),
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-- --------------------------------------------------------

--
-- Table structure for table `content_field_right_related`
--

CREATE TABLE IF NOT EXISTS `content_field_right_related` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_right_related_nid` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`),
  KEY `field_right_related_nid` (`field_right_related_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_field_supplement`
--

CREATE TABLE IF NOT EXISTS `content_field_supplement` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_supplement_fid` int(11) DEFAULT NULL,
  `field_supplement_list` tinyint(4) DEFAULT NULL,
  `field_supplement_data` text,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_supplement`
--

INSERT INTO `content_field_supplement` (`vid`, `nid`, `delta`, `field_supplement_fid`, `field_supplement_list`, `field_supplement_data`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `content_field_table`
--

CREATE TABLE IF NOT EXISTS `content_field_table` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `delta` int(10) unsigned NOT NULL DEFAULT '0',
  `field_table_fid` int(11) DEFAULT NULL,
  `field_table_list` tinyint(4) DEFAULT NULL,
  `field_table_data` text,
  PRIMARY KEY (`vid`,`delta`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_table`
--

INSERT INTO `content_field_table` (`vid`, `nid`, `delta`, `field_table_fid`, `field_table_list`, `field_table_data`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `content_field_teaser`
--

CREATE TABLE IF NOT EXISTS `content_field_teaser` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_teaser_value` longtext,
  `field_teaser_format` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_field_teaser`
--

INSERT INTO `content_field_teaser` (`vid`, `nid`, `field_teaser_value`, `field_teaser_format`) VALUES
(15, 15, NULL, NULL),
(18, 15, NULL, NULL),
(19, 15, NULL, NULL),
(29, 26, NULL, NULL),
(34, 31, NULL, NULL),
(35, 32, NULL, NULL),
(36, 33, NULL, NULL),
(41, 38, '\r\n', 2);

-- --------------------------------------------------------

--
-- Table structure for table `content_group`
--

CREATE TABLE IF NOT EXISTS `content_group` (
  `group_type` varchar(32) NOT NULL DEFAULT 'standard',
  `type_name` varchar(32) NOT NULL DEFAULT '',
  `group_name` varchar(32) NOT NULL DEFAULT '',
  `label` varchar(255) NOT NULL DEFAULT '',
  `settings` mediumtext NOT NULL,
  `weight` int(11) NOT NULL DEFAULT '0',
  PRIMARY KEY (`type_name`,`group_name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_group`
--

INSERT INTO `content_group` (`group_type`, `type_name`, `group_name`, `label`, `settings`, `weight`) VALUES
('standard', 'article', 'group_page', 'Page Number', 'a:2:{s:4:"form";a:2:{s:5:"style";s:8:"fieldset";s:11:"description";s:0:"";}s:7:"display";a:9:{s:6:"weight";s:2:"25";s:5:"label";s:5:"above";s:6:"teaser";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:4:"full";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:2;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:1;}i:3;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:1;}i:4;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:1;}s:11:"description";s:0:"";s:5:"token";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}}}', 8),
('standard', 'conference_paper', 'group_manu', 'MANUSCRIPT FILES', 'a:2:{s:4:"form";a:2:{s:5:"style";s:8:"fieldset";s:11:"description";s:0:"";}s:7:"display";a:9:{s:6:"weight";s:1:"7";s:5:"label";s:5:"above";s:6:"teaser";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:4:"full";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:11:"description";s:0:"";i:4;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:2;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:3;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:5:"token";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}}}', 7),
('standard', 'editors_choice', 'group_ec_featured_nodes', 'Featured Nodes', 'a:2:{s:4:"form";a:2:{s:5:"style";s:8:"fieldset";s:11:"description";s:76:"Note: if any custom HTML is entered this referenced content will be ignored.";}s:7:"display";a:8:{s:11:"description";s:0:"";s:6:"teaser";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:4:"full";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:4;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:2;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}i:3;a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:5:"token";a:2:{s:6:"format";s:8:"fieldset";s:7:"exclude";i:0;}s:5:"label";s:5:"above";}}', -2);

-- --------------------------------------------------------

--
-- Table structure for table `content_group_fields`
--

CREATE TABLE IF NOT EXISTS `content_group_fields` (
  `type_name` varchar(32) NOT NULL DEFAULT '',
  `group_name` varchar(32) NOT NULL DEFAULT '',
  `field_name` varchar(32) NOT NULL DEFAULT '',
  PRIMARY KEY (`type_name`,`group_name`,`field_name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_group_fields`
--

INSERT INTO `content_group_fields` (`type_name`, `group_name`, `field_name`) VALUES
('article', 'group_page', 'field_article_no'),
('article', 'group_page', 'field_page'),
('conference_paper', 'group_manu', 'field_abstract'),
('conference_paper', 'group_manu', 'field_cover'),
('conference_paper', 'group_manu', 'field_images'),
('conference_paper', 'group_manu', 'field_maindoc'),
('conference_paper', 'group_manu', 'field_supplement'),
('conference_paper', 'group_manu', 'field_table'),
('conference_paper', 'group_manu', 'field_title'),
('editors_choice', 'group_ec_featured_nodes', 'field_ec_featured_nodes');

-- --------------------------------------------------------

--
-- Table structure for table `content_node_field`
--

CREATE TABLE IF NOT EXISTS `content_node_field` (
  `field_name` varchar(32) NOT NULL DEFAULT '',
  `type` varchar(127) NOT NULL DEFAULT '',
  `global_settings` mediumtext NOT NULL,
  `required` tinyint(4) NOT NULL DEFAULT '0',
  `multiple` tinyint(4) NOT NULL DEFAULT '0',
  `db_storage` tinyint(4) NOT NULL DEFAULT '1',
  `module` varchar(127) NOT NULL DEFAULT '',
  `db_columns` mediumtext NOT NULL,
  `active` tinyint(4) NOT NULL DEFAULT '0',
  `locked` tinyint(4) NOT NULL DEFAULT '0',
  PRIMARY KEY (`field_name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_node_field`
--

INSERT INTO `content_node_field` (`field_name`, `type`, `global_settings`, `required`, `multiple`, `db_storage`, `module`, `db_columns`, `active`, `locked`) VALUES
('field_abstract', 'filefield', 'a:3:{s:10:"list_field";s:1:"0";s:12:"list_default";i:1;s:17:"description_field";s:1:"0";}', 1, 0, 1, 'filefield', 'a:3:{s:3:"fid";a:3:{s:4:"type";s:3:"int";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"list";a:4:{s:4:"type";s:3:"int";s:4:"size";s:4:"tiny";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"data";a:3:{s:4:"type";s:4:"text";s:9:"serialize";b:1;s:5:"views";b:1;}}', 1, 0),
('field_abstrak', 'text', 'a:4:{s:15:"text_processing";s:1:"1";s:10:"max_length";s:0:"";s:14:"allowed_values";s:0:"";s:18:"allowed_values_php";s:0:"";}', 0, 0, 0, 'text', 'a:2:{s:5:"value";a:5:{s:4:"type";s:4:"text";s:4:"size";s:3:"big";s:8:"not null";b:0;s:8:"sortable";b:1;s:5:"views";b:1;}s:6:"format";a:4:{s:4:"type";s:3:"int";s:8:"unsigned";b:1;s:8:"not null";b:0;s:5:"views";b:0;}}', 1, 0),
('field_article_no', 'number_integer', 'a:6:{s:6:"prefix";s:0:"";s:6:"suffix";s:0:"";s:3:"min";s:0:"";s:3:"max";s:0:"";s:14:"allowed_values";s:0:"";s:18:"allowed_values_php";s:0:"";}', 0, 0, 1, 'number', 'a:1:{s:5:"value";a:3:{s:4:"type";s:3:"int";s:8:"not null";b:0;s:8:"sortable";b:1;}}', 1, 0),
('field_article_nor', 'text', 'a:4:{s:15:"text_processing";s:1:"0";s:10:"max_length";s:0:"";s:14:"allowed_values";s:0:"";s:18:"allowed_values_php";s:0:"";}', 0, 0, 1, 'text', 'a:1:{s:5:"value";a:5:{s:4:"type";s:4:"text";s:4:"size";s:3:"big";s:8:"not null";b:0;s:8:"sortable";b:1;s:5:"views";b:1;}}', 1, 0),
('field_audio_file', 'filefield', 'a:3:{s:10:"list_field";b:0;s:12:"list_default";i:1;s:17:"description_field";s:1:"0";}', 0, 0, 1, 'filefield', 'a:3:{s:3:"fid";a:3:{s:4:"type";s:3:"int";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"list";a:4:{s:4:"type";s:3:"int";s:4:"size";s:4:"tiny";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"data";a:3:{s:4:"type";s:4:"text";s:9:"serialize";b:1;s:5:"views";b:1;}}', 1, 0),
('field_author_photo', 'filefield', 'a:3:{s:10:"list_field";s:1:"0";s:12:"list_default";i:1;s:17:"description_field";s:1:"0";}', 0, 0, 1, 'filefield', 'a:3:{s:3:"fid";a:3:{s:4:"type";s:3:"int";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"list";a:4:{s:4:"type";s:3:"int";s:4:"size";s:4:"tiny";s:8:"not null";b:0;s:5:"views";b:1;}s:4:"data";a:3:{s:4:"type";s:4:"text";s:9:"serialize";b:1;s:5:"views";b:1;}}', 1, 0),
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-- --------------------------------------------------------

--
-- Table structure for table `content_node_field_instance`
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CREATE TABLE IF NOT EXISTS `content_node_field_instance` (
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--
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('field_op_author', 'blog', -4, 'Author(s)', 'nodereference_autocomplete', 'a:4:{s:18:"autocomplete_match";s:8:"contains";s:4:"size";s:2:"60";s:13:"default_value";a:1:{i:0;a:2:{s:3:"nid";N;s:14:"_error_element";s:50:"default_value_widget][field_op_author][0][nid][nid";}}s:17:"default_value_php";N;}', 'a:9:{s:6:"weight";s:2:"-4";s:6:"parent";s:0:"";i:4;a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:1;}s:5:"label";a:1:{s:6:"format";s:5:"above";}s:6:"teaser";a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:1;}s:4:"full";a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:1;}i:2;a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:0;}i:3;a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:0;}s:5:"token";a:2:{s:6:"format";s:7:"default";s:7:"exclude";i:0;}}', '', 'nodereference', 1),
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INSERT INTO `content_node_field_instance` (`field_name`, `type_name`, `weight`, `label`, `widget_type`, `widget_settings`, `display_settings`, `description`, `widget_module`, `widget_active`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `content_type_article`
--

CREATE TABLE IF NOT EXISTS `content_type_article` (
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  KEY `nid` (`nid`),
  KEY `field_mid_nid` (`field_mid_nid`),
  KEY `field_issue_nid` (`field_issue_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_type_article`
--

INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
(346, 343, 'Relationship between Central Corneal Thickness and Severity of Open Angle Glaucoma using Optical Coherence Tomography', 'Dr Mushawiahti  Mustapha,  Department  of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar  Tun  Razak,  56000 Cheras,  Kuala  Lumpur.  \r\nTel:  +603-91455981 Fax:  +60391737836  \r\nEmail: drmusha@yahoo.com/ drmusha@gmail.com', NULL, 344, 107, 113, NULL),
(353, 350, 'Primary Adenocarcinoma of the Fallopian Tube  Mimicking Pelvic Inflammatory Disease', 'Prof Mohammad Rafaee Tamby, Department of O&G, Faculty of Medicine and Health Sciences, University Putra Malaysia, 43400 UPM, Serdang Selangor Darul Ehsan. \r\nTel: 03-89472640 Fax: 03-89472639 \r\nEmail: rafaee.tamby@gmail.com ', NULL, 344, 139, 142, NULL),
(360, 357, 'Early Lineage Switch from T-Acute Lymphoblastic Leukaemia to Common B-All', 'Dr Hafiza Alauddin, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Kuala Lumpur. \r\nTel: 603-9145 5837/5357. Fax: 603- 91456676. \r\nEmail: drhafiza@ppukm.ukm.my ', NULL, 344, 131, 138, NULL),
(368, 365, 'Breast Self Examination: To Do or Not To Do?', 'Leelavathi Muthupalaniappen, Department of Family Medicine, Medical Faculty, Hospital University Kebangsaan Malaysia (HUKM), Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. \r\nEmail: drleela@mail.hukm.ukm.my', NULL, 332, 1, 4, NULL),
(369, 366, 'BCR/ABL Gene in Chronic Myeloid Leukaemia', 'Dr Reena Rahayu Md Zin, Cytogenetics Unit, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. \r\nEmail: ayureena@hotmail.com', NULL, 332, 5, 13, NULL),
(370, 367, 'Patient Satisfaction with Quality Nursing Care', 'Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. \r\nEmail: ho@mail.hukm.ukm.my', NULL, 332, 14, 19, NULL),
(371, 368, NULL, 'Assoc. Professor (Clinical) Dr. Jaafar Md.Zain, Department of Anaesthesiology & Intensive care, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Bandar Tun Razak Cheras, Kuala Lumpur Malaysia. \r\nEmail: Jaafar@mail.hukm.ukm.my', NULL, 332, 20, 24, NULL),
(372, 369, 'Prevalence of Non-Dengue Thrombocytopaenia', 'Dr.  Tong  Seng  Fah,  Department  of  Family  Medicine, Faculty of Medicine, National University of Malaysia (UKM), Jalan Yaacob Latif, Cheras, 56000 Kuala Lumpur, Malaysia.\r\nE-mail: tsf@mail.hukm.ukm.my ', NULL, 332, 25, 30, NULL),
(373, 370, 'Importance of Knowing Potential for Cure', 'Raja Lexshimi Raja Gopal, \r\nNursing Department, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latiff,  Bandar Tun Razak, 56000, CHERAS, KUALA LUMPUR. ', NULL, 332, 31, 35, NULL),
(374, 371, NULL, 'Phan Chin Lee, \r\nHaematology Unit, Genetic - Cytogenetic Laboratory, CaRC, Institute for Medical Research Malaysia, Jalan Pahang 50588, Kuala Lumpur. E-mail address: cuapcl@hotmail.com', NULL, 332, 36, 44, NULL),
(375, 372, 'Conventional & Molecular Cytogenetics in Microdeletion Syndrome', 'Salwati Shuib, Cytogenetic Unit, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. Email: salwati@mail.hukm.ukm.my', NULL, 332, 45, 52, NULL),
(376, 373, 'Biphenotypic Acute Leukemia', 'Dr. Raja Zahratul Azma bt. Raja Sabudin, \r\nDepartment of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia (UKM), Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia. \r\nE-mail: zahratul@mail.hukm.ukm.my', NULL, 332, 53, 60, NULL),
(377, 374, NULL, 'Dr Asma Abdullah, Department of Otorhinolaryngology, Medical Faculty, University Kebangsaan Malaysia, Jalan Yaakub Latif, 56000 Cheras, Kuala Lumpur, Malaysia. \r\nE-mail: asmaent@yahoo.com', NULL, 332, 61, 66, NULL),
(378, 375, 'HPV, p53 & c-erbB-2 in Juvenile Laryngeal Papillomatosis', 'Profesor Madya Dr Siti Aishah Md Ali, Jabatan Patologi,  Fakulti Perubatan, Universiti Kebangsaan Malaysia, Jalan Yaakob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nE-mel: saishah@mail.hukm.ukm.my', NULL, 332, 67, 74, NULL),
(379, 376, 'FNAC of Metastatic Renal Cell Carcinoma', 'Associate Professor Dr Wan Muhaizan bt Wan Mustaffa, Department of Pathology, Faculty of Medicine, University Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000, Cheras, Kuala Lumpur, Malaysia. \r\ne-mail : muhaizan@mail.hukm.ukm.my', NULL, 332, 75, 80, NULL),
(380, 377, NULL, NULL, NULL, 332, 81, 84, NULL),
(381, 378, 'Villoglandular Papillary Adenocarcinoma of the Uterine Cervix', NULL, NULL, 332, 85, 90, NULL),
(382, 379, NULL, 'Dr Mae-Lynn Catherine Bastion, Department of Ophthalmology,Hospital Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, 56000 Kuala Lumpur, Malaysia. \r\nEmail: maelynnbdr@hotmail.com', NULL, 332, 91, 93, NULL),
(383, 380, 'Hepatitis B Virus Genotypes and Mutations', 'Dr. Ton So Har, School of Arts and Sciences, Monash University Malaysia, Petaling Jaya 46150, Malaysia.  \r\nEmail: ton.so.ha@artsci.monash.edu.my', NULL, 333, 1, 25, NULL),
(384, 381, NULL, 'Ho Siew Eng, \r\nDepartment of Nursing, Faculty of Medicine, \r\nUniversiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   \r\nEmail: ho@mail.hukm.ukm.my', NULL, 333, 26, 33, NULL),
(385, 382, 'Anxiety & Depression among High Risk Pregnant Women', 'Raja Lexshimi Raja Gopal , Nursing Department, Faculty of Medicine, Universiti  Kebangsaan Malaysia, Jalan Yaacob Latiff, Bandar Tun Razak, 56000, Cheras, Kuala Lumpur.  \r\nEmail:  lex@mail.hukm. ukm.my', NULL, 333, 34, 41, NULL),
(386, 383, 'Effects of Dysmenorrhoea on School Activities', 'Dr Verna Lee Kar Mun, Department of Family Medicine Faculty of Medicine, Universiti Kebangsaan Malaysia,  Jalan Yaacob Latif,  Bandar Tun Razak,  56000 Cheras, Kuala Lumpur.  \r\nEmail: verna@mail.hukm.ukm.my', NULL, 333, 42, 47, NULL),
(387, 384, 'Female Sexual Dysfunction', 'Clinical Associate Professor Dr. Hatta Sidi, \r\nDepartment of Psychiatry, Universiti Kebangsaan Malaysia (UKM), Jalan Yaakob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: hattasidi@hotmail.com', NULL, 333, 48, 57, NULL),
(388, 385, NULL, 'Professor Dr. Sharifah Noor Akmal Syed Husain, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   \r\nEmail: sharifah@mail.hukm.ukm.my', NULL, 333, 58, 65, NULL),
(389, 386, 'Ovarian Volume, Uterine Size and Endometrial Thickness', 'Professor Nafisah Adeeb, Dept of Obstetrics & Gynaecology, Medical Faculty, Universiti Kebangsaan Malaysia,  Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur.  \r\nEmail:naadeeb@mail.hukm.ukm.my', NULL, 333, 66, 79, NULL),
(390, 387, 'Smoking among Upper Secondary Schoolboys', 'Assoc. Prof. Dr. Khairani Omar, Department of Family Medicine,Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur.  \r\nEmail : khairani@mail.hukm.ukm.my', NULL, 333, 80, 85, NULL),
(391, 388, 'Sharps Injury', 'Santhna Letchmi Panduragan, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latiff, 56000 Cheras, Kuala Lumpur.  \r\nEmail:  santhna@mail.hukm.ukm.my', NULL, 333, 86, 92, NULL),
(392, 389, 'Work Stress among Management Staff', 'Prof. Dr. Noor Hassim Ismail, \r\nDepartment of Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latiff, 56000 Cheras, Kuala Lumpur.  \r\nEmail: hassim@mail.hukm.ukm.my', NULL, 333, 93, 98, NULL),
(393, 390, NULL, 'Professor Dr. Fadilah S. Abdul Wahid, \r\nDepartment of Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   \r\nEmail: sfadilah@mail.hukm.ukm.my', NULL, 333, 99, 102, NULL),
(394, 391, NULL, 'Muhd Helmi Azmi, Department of Anaesthesiology and Intensive Care, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur.Email:mdhelmi2001@yahoo.co.uk', NULL, 334, 103, 109, NULL),
(395, 392, 'Knowledge, Perception of Skills and Confidence Level in Performing CPR', 'Dr Ismail Mohd Saiboon. Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. \r\nTel: 03-91702020 Fax; 603- 91738154 \r\nE-mail: ismail@mail.hukm.ukm.my', NULL, 334, 110, 116, NULL),
(396, 393, 'Home Care Nursing Program', 'Samsiah Mat, \r\nDepartment of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail:  samsiah@mail.hukm.ukm.my', NULL, 334, 117, 124, NULL),
(397, 394, 'p53 Expression in Cervical Neoplasms', 'Dr Tan Geok Chin, \r\nDepartment of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia. Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia.  \r\nEmail: tan_geok_chin@yahoo.com', NULL, 334, 125, 132, NULL),
(398, 395, NULL, 'Associate Prof Dr Asma bt Abdullah, Department of Otorhinolaringology, Faculty of Medicine, University Kebangsaan Malaysia, Jalan Yaacob Latif, 56000 Cheras, Kuala Lumpur.', NULL, 334, 133, 138, NULL),
(399, 396, 'Placenta Praevia', 'Dr M I Nor Azlin. Dept. of Obstetric & Gynaecology Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. \r\nTel: 03-91455949 Fax; 603- 91738946 E-mail: azlinm@mail.hukm.ukm.my', NULL, 334, 139, 145, NULL),
(400, 397, 'Prevalence of Stress and Coping Mechanism', 'Dr M I Nor Azlin. Dept. of Obstetric & Gynaecology Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000 Kuala Lumpur. Tel: 03-91455949 Fax; 603- 91738946 E-mail: azlinm@mail.hukm.ukm.my', NULL, 334, 146, 153, NULL),
(401, 398, 'Night Eating Syndrome with Morbid Obesity and Dysthymia', 'Associate Professor Dr Ainsah Omar, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   Email: ainsah@mail.hukm.ukm.my / oyjaao@yahoo.com.my', NULL, 334, 154, 157, NULL),
(402, 399, 'Parapharyngeal Abscess in Immunocompromised Patients', 'Dr Norhaslinda Abdul Gani , Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia. Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia.  \r\nEmail: haslinda.gani@yahoo.com', NULL, 334, 158, 163, NULL),
(403, 400, 'Pleomorphic Adenoma Originating from Submandibular Salivary Gland', 'Dr Norhaslinda Abdul Gani , Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia. Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia.  \r\nEmail: haslinda.gani@yahoo.com', NULL, 334, 164, 168, NULL),
(404, 401, 'Further Rehabilitation in Longer-Term Stroke Patients', 'Dr Noor Azah Aziz, Department of Family Medicine UKM, Jalan  Yaacob  Latiff,  Bandar  Tun  Razak,  56000  Cheras,  Kuala Lumpur.  Tel:  03  91733333   samb   2846  \r\nFax: 03 91738153 \r\nEmail: azah@mail.hukm.ukm.my / a.azah@yahoo.co.uk\r\n', NULL, 335, 1, 6, NULL),
(405, 402, 'Patients’ Satisfaction with the Emergency Department', 'Dr Ismail Saiboon, Department of Emergency Medicine, Hospital Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: ismail@mail.hukm.ukm.my', NULL, 335, 7, 13, NULL),
(406, 403, 'Quality of Life among Patients with Schizophrenia', 'Associate Professor Dr Ainsah Omar, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   \r\nEmail: ainsah@mail.hukm.ukm.my / oyjaao@yahoo.com.my', NULL, 335, 14, 21, NULL),
(407, 404, 'Y Chromosomal Material in Turner Syndrome', 'Dr Reena Rahayu Md Zin, Cytogenetics Unit, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, Kuala Lumpur.\r\nEmail: ayureena@hotmail.com', NULL, 335, 22, 29, NULL),
(408, 405, 'Information on Breastfeeding among Pregnant Mothers', 'Assoc. Prof Dr Tan Ay Eeng, Department of Obstetrics & Gynecology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: ae_tan2002@yahoo.com.au', NULL, 335, 30, 37, NULL),
(409, 406, 'Prevalence and Risk Factors of WRULD', 'Professor Dr. Noor Hassim Ismail, Department Of Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur.   \r\nEmail:  hassim@mail.hukm.ukm.my', NULL, 335, 38, 45, NULL),
(410, 407, 'Recollections Expressed by Mechanically Ventilated Patients of ICU', 'Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Kuala Lumpur, Malaysia.   \r\nEmail: ho@mail.hukm.ukm.my', NULL, 335, 46, 53, NULL),
(411, 408, 'Medical Graduates’ Perception of their Communication Skills', 'Dr Abdus Salam, Department of Medical Education, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: salam@mail.hukm.ukm.my', NULL, 335, 54, 58, NULL),
(412, 409, 'Utility of Pap Smears and Mammograms', 'Professor Dr Nafisah Adeeb, Department of Obstetrics & Gynaecology, Faculty of Medicine, Universiti Kebangsaan Malaysia. Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia.  Email:naadeeb@mail.hukm.ukm.my', NULL, 335, 59, 68, NULL),
(413, 410, 'Anaplastic Large Cell Lymphoma', 'Professor Dr Siti Aishah Md Ali, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kualal Lumpur. \r\nEmail: saishah@mail.hukm.ukm.my', NULL, 335, 69, 74, NULL),
(414, 411, 'Clinical Pathway to Improve Healthcare Quality', 'Dr. Aniza Ismail, Department of Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: aniza.ismail@gmail.com', NULL, 337, 239, 246, NULL),
(415, 412, 'Effects of tocotrienols on ovariectomised-rat bone', 'Professor Dr Ima Nirwana Soelaiman. Department of Pharmacology, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur. Tel: 03-92897281 / 5514 Fax; 603- 26938205 \r\nE-mail: imasoel@medic.ukm.my', NULL, 337, 247, 255, NULL),
(416, 413, 'Gamma-tocotrienol Alters Protein Expression of HepG2 Cell Line', 'Dr Zakiah Jubri @ Mohd Zufri, Department of Biochemistry, Faculty of Medicine, Universiti Kebangsaan Malaysia Kuala Lumpur, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur, Malaysia. \r\nEmail: zakiah@medic.ukm.my', NULL, 337, 256, 262, NULL),
(417, 414, 'Zingiber officinale Roscoe and apoptosis', 'Professor Dr. Yasmin Anum Mohd Yusof, Department of Biochemistry, Universiti Kebangsaan Malaysia Medical Centre (UKMMC), Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur. \r\nTel: 603-9289 5297; Fax: 603-2693 8037. Email: anum@medic.ukm.my', NULL, 337, 263, 274, NULL),
(418, 415, 'Great Teacher Creates Effective Learning Environment', 'Mohd Said Nurumal, Kulliyyah (Faculty) of Nursing, International Islamic University Malaysia (IIUM), P.0 Box 141, 25710 Kuantan, Pahang Darul Makmur. \r\nTel: 09-5133710. Fax: 09-5133615. Email: mohdsaid@iiu.edu.my ', NULL, 337, 275, 280, NULL),
(419, 416, 'Information needs of post MI patients', 'Ho Siew Eng, Department of Nursing, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: ho@ppukm.ukm.my', NULL, 337, 281, 287, NULL),
(420, 417, 'Venofer for Treatment of Iron Deficiency Anaemia In Pregnancy', 'Assoc. Prof Dr Tan Ay Eeng, Department of Obstetrics & Gynecology, UKM Medical Centre, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: ae_tan2002@yahoo.com.au', NULL, 337, 288, 293, NULL),
(421, 418, 'MRSA Infection in Vascular Surgical Patients', 'Associate Professor Dr. Ismail Sagap, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: drisagap @mail.hukm.ukm.my', NULL, 337, 294, 299, NULL),
(422, 419, NULL, 'Associate Professor Dr. Ahmad Nazlim Yusoff, Program Pengimejan Diagnostik & Radioterapi, Fakulti Sains Kesihatan Bersekutu Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz 50300 Kuala Lumpur.  Email: nazlim@medic.ukm.my ', NULL, 337, 300, 317, NULL),
(423, 420, 'Polypharmacy, an option prior to Clozapine?', 'Dr. Amer Siddiq Amer Nordin, Department of Psychological Medicine, Faculty of Medicine, University Malaya, 50603 Kuala Lumpur. \r\nTel: 03-79492068. Fax: 03-79556477. Email: amersiddiq@um.edu.my', NULL, 337, 318, 321, NULL),
(424, 421, 'Mothers’ Breast Feeding Practice & Knowledge', 'Assoc Prof Dr Khairani Omar, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455555. Fax: 603-91738153. Email: khairani@ppukm.ukm.my', NULL, 12, 1, 7, NULL),
(425, 422, NULL, 'Prof. Dr. Musalmah Mazlan, Department of Biochemistry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abd. Aziz, 50300 Kuala Lumpur. \r\nTel: 603-92897222. Fax : 603-26938037. Email: musalmah@medic.ukm', NULL, 12, 8, 15, NULL),
(426, 423, 'Knowledge, attitude and practice of nurses in medication administration', 'Raja Lexshimi Raja Gopal, Department of Nursing, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nEmail: lex@ppukm.ukm.my', NULL, 12, 16, 24, NULL),
(427, 424, 'Tocotrienol rich fraction inhibits melanin synthesis', 'Assoc Prof Dr. Suzana Makpol, Department of Biochemistry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abd. Aziz, 50300 Kuala Lumpur. Tel: 603-92897296/7222. Fax: 603-26938037. Email: suzana@medic.ukm.my', NULL, 12, 25, 34, NULL),
(428, 425, 'Patient Satisfaction on Waiting Time', ':  Raja Lexshimi Raja Gopal, Department of Nursing, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Email: lex@ppukm.ukm.my', NULL, 12, 35, 46, NULL),
(429, 426, 'Knowledge & attitude in post operative pain management', 'Ho Siew Eng, Department of Nursing, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Email: ho@ppukm.ukm.my', NULL, 12, 47, 52, NULL),
(430, 427, 'Waiting Times For Elective Orthopaedic Surgeries', 'Dr. Azimatun Noor Bt Aizuddin, Department of Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 6012-696 3215. Email: azimatunnoor@gmail.com', NULL, 12, 53, 60, NULL),
(431, 428, 'Coagulase negative staphylococcus necrotizing pneumonia', 'Professor Dr. Cheah Fook Choe, Department of Paediatrics, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-9145 5391. Fax: 603-9173 7827. Email: cheahfc@ppukm.ukm.my', NULL, 12, 61, 63, NULL),
(432, 429, 'Role of ABCB1 C3435T Variant in Epilepsy', 'Prof Dr Raymond Azman Ali, Department of Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Email: raymond@ppukm.ukm.my', NULL, 111, 64, 75, NULL),
(433, 430, 'Dental Students’ Knowledge and Perception of Elderly', 'Dr Tanti Irawati Rosli, Department of Dental Public Health, Faculty of Dentistry, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur. Tel: 603-9289 7750. Fax: 603-9289 7845. Email: tantiir@hotmail.com', NULL, 111, 76, 83, NULL),
(434, 431, 'Retropharyngeal Abscess', 'Dr. Marina Bt Mat Baki, Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Fax: 603-91737840. Email: marinadrent@gmail.com', NULL, 111, 84, 90, NULL),
(435, 432, 'Riverine Communities and Waterborne Diseases', 'Utharas Arumugam, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-9171 6265. Fax : 603-9173 8254. Email: utharas@usa.net', NULL, 111, 91, 100, NULL),
(436, 433, 'Stress and coping styles in nurses', 'Dr. Wan Salwina Wan Ismail, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91456158. Fax: 603-91737841. Email: wan@ppukm.ukm.my', NULL, 111, 101, 107, NULL),
(437, 434, 'Role of powered instrumentation in antrochoanal polyps', 'Prof Dato’ Dr Balwant Singh Gendeh, Department of Othorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Email: bsgendeh@gmail.com', NULL, 111, 108, 114, NULL),
(438, 435, 'Workplace Violence Experienced by Nurses', 'Hamidah Hassan, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91456250. Email: midah@ppukm.ukm.my', NULL, 111, 115, 121, NULL),
(439, 436, 'Diagnosis of Langerhans cell sarcoma', 'Associate Professor Dr. Noraidah Masir, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455357. Fax: 603-91737340. Email: noraidah@ppukm.ukm.my', NULL, 111, 122, 126, NULL),
(440, 437, 'Intolerance to radiotherapy in squamous cell carcinoma of the tongue', 'Assoc Prof Dr Hamidah Alias, Department of Pediatrics, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455396. Fax: 603-91737827. Email: midalias@ppukm.ukm.my', NULL, 111, 133, 137, NULL),
(441, 438, 'Metaplastic Breast Carcinoma', 'Prof Dr Rohaizak Muhammad, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455002. Fax: 603-91737831. Email: rohaizak@ppukm.ukm.my', NULL, 111, 127, 132, NULL),
(442, 439, 'Chlorella vulgaris and expression of COL and MMP genes', 'Assoc. Prof. Dr. Suzana Makpol, Department of Biochemistry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur. Tel: 603-9289 7296 / 7222. Fax: 603-2693 8037. Email: suzanamakpol@yahoo.com', NULL, 252, 1, 12, NULL),
(443, 440, 'Helicobacter pylori infection among patients with dyspepsia', 'Alfizah Hanafiah, Department of Medical Microbiology & Immunology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455932. Fax: 603-91737336. Email: alfizah @ppukm.ukm.my', NULL, 252, 13, 21, NULL),
(444, 441, 'MRD status in childhood ALL by flow cytometry', 'Dr Raja Zahratul Azma Raja Sabudin, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455780. Fax: 603-91736676. Email: zahratul@ppukm.ukm.my', NULL, 252, 22, 33, NULL),
(445, 442, 'Peripheral neuropathy and Type II diabetes', 'Assoc Prof Dr Noor Azah Aziz, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-9145 6116. Fax: 603-9173 8153. Email: azah@ppukm.ukm.my', NULL, 252, 34, 40, NULL),
(446, 443, 'Acute Mastoid Abscess', 'Assoc Prof Dr Asma Abdullah. Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Fax: 603-91737840. Email: asmaent@yahoo.com.my', NULL, 252, 41, 44, NULL),
(447, 444, 'Concomitant t(8;21) and trisomy 4 in AML', 'Phan Chin Lee, Clinical Hematology Laboratory, Department of Hematology, Hospital Ampang, Jalan Mewah Utara, Pandan Mewah, 68000 Ampang, Selangor. Tel: 603-4289 6532 / 6226 / 6219. Fax: 603-4295 4666. Email: cuapcl@hotmail.com', NULL, 252, 45, 48, NULL),
(448, 445, 'Invasive thymoma in children', 'Assoc. Prof. Dr. A. Hamidah, Department of Paediatrics, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91456637. Fax: 603-91737827. Email: midalias@ppukm.ukm.my', NULL, 252, 49, 54, NULL),
(449, 446, 'Oncocytic Papilloma', 'Dr Mazita Ami, Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91456045. Fax: 603-91737840. Email: mazitaami@yahoo.com', NULL, 252, 55, 59, NULL),
(450, 447, 'Ascariasis among Orang Asli Children', 'Prof. Dr. Mohamed Kamel Abd Ghani, Department of Biomedical Science, Faculty of Health Science, Universiti Kebangsaan Malaysia, 50300 Jalan Raja Muda Abdul Aziz, Kuala Lumpur. Tel: 603-92897634. Fax: 603-26929032. Email: mkamal@medic.ukm.my', NULL, 329, 60, 65, NULL),
(451, 448, 'HPV genotypes in cervical cancer', 'Prof Dato'' Dr Syed Mohamed Aljunid, United Nations University-International Institute of Global Health (UNU-IIGH), UKM Medical Centre, Jalan Yaacob Latiff,    56000 Cheras Kuala Lumpur. Tel: +603-9171 5394. Fax: +603- 9171 5402. ', NULL, 329, 66, 76, NULL),
(452, 449, 'Quality of life in post coronary bypass patients', 'Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-9145 6262. Fax: 603-9173 8254. Email: ho@ppukm.ukm.my', NULL, 329, 77, 85, NULL),
(453, 450, 'Facial nerve schwannomas', 'Dr Mazita Ami, Department of Otorhinolaryngology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras Kuala Lumpur. Tel: 603-91456046. Fax: 603-91456675. Email: mazitaami@yahoo.com', NULL, 329, 86, 92, NULL),
(454, 451, 'Adjunctive ranibizumab for diabetic vitrectomy', 'Assoc Prof Dr Mae-Lynn Catherine Bastion, Department of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 603-91455981. Fax: 603-91456673. Email: maelynnb2003@yahoo.com', NULL, 329, 93, 102, NULL),
(455, 452, 'Extensive congenital cholesteatoma', 'Dr Roslenda bt Abdul Rahman, Department of ORL Head & Neck Surgery, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel:+603 9145 6576. Fax: +603 9145 7765. Email: lenda_orl@yahoo.com', NULL, 329, 103, 107, NULL),
(456, 453, 'Cri-du-chat syndrome: Application of array CGH', 'Assoc. Prof. Dr. Zarina Abdul Latif, Department of Paediatrics, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nTel: 603-91457888. Fax: 603-91456637. Email: zarinaal@ppukm.ukm.my', NULL, 329, 108, 113, NULL),
(457, 454, 'Primary thyroid lymphoma', 'Dr Azlin Ithnin, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nTel: 603-9145 5837. Fax: 603-9173 6676. Email: nazlin@ppukm.ukm.my', NULL, 329, 114, 119, NULL),
(458, 455, 'Effects of phytosterol on lipid peroxidation', 'Dr. Nur Azlina  Mohd Fahmi,  Department  of Pharmacology,  Faculty  of  Medicine,  Universiti  Kebangsaan  Malaysia,  Jalan  Raja  Muda Abd  Aziz,  50300, Kuala Lumpur. Tel: 603-92897281. Fax: 603-26938205. Email: nazlina@medic.ukm.my ', NULL, 328, 25, 32, NULL),
(459, 456, 'Genetic markers in breast cancer', 'Dr.  Kelly  Avery-Kiejda,  Hunter  Medical  Research Institute,  Room  3644,  Level  3,  John  Hunter  Hospital  Lookout  Rd.  New Lambton  Heights,  NSW,  2305, Australia \r\nTel: +61 2 4985 5663. Fax: +61 2 4985 5895. Email: Kelly.Kiejda@newcastle.edu.au', NULL, 328, 1, 24, NULL),
(460, 457, 'CD151 expression in breast carcinoma', 'Ivyna  Bong  Pau  Ni,  Hematology  Unit,  Cancer Research Centre, Institute for Medical Research, Jalan Pahang, 50588 Kuala Lumpur, Malaysia. Tel: +603 2616 2724. Fax: +603 26162707. Email: ivyna@imr.gov.my', NULL, 328, 33, 40, NULL),
(461, 458, 'Level of Recall about Orthodontic Treatment', 'Amy Liew Kia Cheen, Faculty of Dentistry, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur. Tel:  603-92897780.  Fax:  603- 92897750 Email: amyliew1@gmail.com ', NULL, 328, 41, 48, NULL),
(462, 459, 'Susceptibility as a predictor of adolescent smoking ', 'Lim Kuang Hock, Epidemiology & Biostatistics Unit, Institute  for  Medical  Research,  Jalan  Pahang,  50588 Kuala  Lumpur.  Tel:  03-26162747/48.  Fax:  03-2691 1682. Email: limkh@imr.gov.my ', NULL, 328, 49, 58, NULL),
(463, 460, NULL, 'Nor Rizan Kamaluddin,  Haematology Unit, Cancer Research  Centre,  Institute  for  Medical  Research,  Jalan  Pahang,  50588 Kuala  Lumpur.  \r\nTel:  03-26162723 Fax: 03-40411513 Email: norizan@imr.gov.my ', NULL, 328, 59, 67, NULL),
(464, 461, 'Effect of centrifugation on coagulation tests', NULL, NULL, 328, 68, 72, NULL),
(465, 462, 'Posterior urethral valve in Down syndrome', 'Faizah Mohd Zaki. Department of Radiology, Universiti Kebangsaan Malaysia Medical Center, Jalan Yaacob Latiff, 56100 Cheras, Kuala Lumpur. Tel: 03-9145 6177. Fax: 03-91456682. Email: drfaizah@ppukm.ukm,my', NULL, 328, 73, 77, NULL),
(466, 463, 'Determinants of Glucose Control among Elderly Diabetics in Kulim', 'Dr.  Noraishah  Jaafar,  Department  of  Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. \r\nTel: 012-4728665. Fax: 03-91737825. Email: noraishah68@gmail.com ', NULL, 344, 78, 85, NULL),
(467, 464, 'Perceived Stress Related to Nursing Education and Its Influence on Nursing Students'' Academic and Clinical Performance.', 'Gurbinder Kaur Jit Singh, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia (UKM), Jalan Yaacob Latif, Bandar Tun Razak, 56000\r\nCheras, Kuala Lumpur. \r\nTel: 603-91456254. Fax: 603-91456683 Email: gurbin@ppukm.ukm.my\r\n', NULL, 344, 86, 97, NULL),
(468, 465, 'Prevalence and Awareness of Lower Urinary Tract Symptoms among Males in the Outpatient Clinics of Universiti Kebangsaan Malaysia Medical Centre', 'Christopher Ho Chee Kong, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: +60126826599. Fax: +60391456684. Email: chrisckho2002@yahoo.com', NULL, 344, 98, 106, NULL),
(470, 467, 'Post-Discharge Structured Rehabilitation', 'Assoc.  Prof.  Dr Noor Azah  Aziz,  Department  of Family  Medicine,  Faculty  of  Medicine,  Universiti  Kebangsaan  Malaysia,  Jalan  Yaacob  Latif,  Bandar  Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: +603 9145 6116. Fax: 03 91456680 Email: azah@ppukm.ukm.my / bila96@gmail.com', NULL, 344, 114, 122, NULL),
(471, 468, NULL, 'Choor Chee  Ken,  Department  of  Medicine,  Pusat Perubatan  Universiti  Kebangsaan  Malaysia,  56000 Kuala  Lumpur.  \r\nTel:  03-91455555  ext  6089 Fax:  03- 91456679 Email: choor123@gmail.com ', NULL, 344, 123, 125, NULL),
(472, 469, 'Apparent Factor IX Inhibitor', 'Dr. Jameela Sathar, Clinical Hematology Laboratory, Department  of  Hematology,  Hospital  Ampang,  Jalan  Mewah Utara,  Pandan  Mewah,  68000 Ampang, Selangor, Malaysia. \r\nTel: +603-4289 6219/6308 Fax: +603-4297 0059. Email: jsathar@hotmail.com ', NULL, 344, 126, 130, NULL),
(475, 472, '7th Malaysia Indonesia Brunei Medical Science Conference ''TOWARDS A HOLISTIC AND INTEGRATIVE APPROACH IN HEALTHCARE''', NULL, NULL, 338, NULL, NULL, NULL),
(476, 473, '4 th  Malaysia Indonesia Brunei Medical Science Conference ''MEDICAL RESEARCH IN THE ‘OMICS’ ERA''', NULL, NULL, 336, NULL, NULL, NULL),
(920, 915, 'BRIM Immobilizer: Design and Development', 'Prof. Dr. Ismail bin Mohd Saiboon, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaakob Latif, Bandar Tun Razak, Cheras, 56000, Kuala Lumpur. Tel: 03-91455491 Fax: 03-91456577. Email: fadzmail69@yahoo.com.my/ ismail@ppukm.ukm.edu.my.', NULL, 909, 1, 11, 'American College of Surgeons Committee on Trauma (ACS). 2008. Advanced Trauma Life Support® program for doctors. 8th Edition. Chicago.\r\nBrenneman, F.D., Katyal, D., Boulanger, B.R., Tile, M. & Redelmeier, D.A. 1997. Long term outcome in open pelvic fractures. J Trauma 42: 773–777.\r\nDickson, K.F. 2000. The acute management of pelvic ring injuries. In: Kellam JF. et al. editors. Orthopedic knowledge update: trauma 2. Rosemont, IL: AAOS.\r\nErtel, W., Keel, M., Eid, K., Platz, A. & Trentz, O. 2001. Control of severe hemorrhage using C-clamp and pelvic packing in multiply injured patients with pelvic ring disruption. J Orthop Trauma 15: 468–474.\r\nFlint, L. & Cryer, H.G. 2010. Pelvic Fracture: The last 50 years. J Trauma 69: 483-488.\r\nHeetveld, M.J., Harris, L., Schlaphoff, G.& Sugrue, M. 2004. Guidelines for the management of haemodynamically unstable pelvic fracture patients. ANZ J Surg 74: 520-529.\r\nHuittinen, V.M. & Slatis, P. 1973. Post mortem angiography and dissection of the hypogastric artery in pelvic fractures. Surgery 73: 454-462.\r\nMcCormack, R., Strauss, EJ., Alwattar, B.J. & Tejwani, N.C. 2010. Diagnosis and management of pelvic fractures. Bull NYU Hosp Jt Dis 68: 281-291.\r\nNational Trauma Database. 2009. Malaysia report. Kuala Lumpur.\r\nPohlemann, T., Bosch, U., Gansslen, A. & Tscherne, H. 1994. The Hannover Experience in management of pelvic fractures. Clin Orthop Relat Res (305): 69-80\r\nRommens, P.M. & Hessmann, M.H. 2002. Staged reconstruction of pelvic ring disruption: differences in morbidity, mortality, radiologic results, and functional outcomes between B1, B2/B3, and C-type lesions. J Orthop Trauma 16: 92–98.\r\nSabariah, F.J., Mahathar, A.W., Fatahul, L.M., Ismail, M.S. (Eds). 2009. National Trauma Database, Malaysia report 2007. Kuala Lumpur Malaysia.\r\nStarr, A.J. 2003. Immediate Management of Pelvic Fractures. Oper Tech Orthop 13: 73-78.\r\nTrikha, V. & Gupta, H. 2011. Current management of pelvic fractures. Jcot 2: 12-18.\r\nTucker, M.C., Nork, S.E., Simonian, P.T. & Routt, M.L. Jr. 2000. Simple anterior pelvic external fixation. J Trauma 49: 989–994.'),
(929, 924, 'Patient Satisfaction in Primary Health Care Clinic', 'Dr. Hizlinda Tohid, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaakob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: 03-91456119. Fax: 603-91456680. Email: hizlinda2202@gmail.com.', NULL, 909, 12, 23, 'Al-Eisa, I., Al-Mutar, M. & Radwan, M. 2005.Patients’ Satisfaction with Primary Health Care Services at Capital Health Region, Kuwait. Middle East Journal of Family Medicine 3(3):10-16.\r\nAngelopoulou, P., Kangis, P. & Babis, G. 1998. Private and public medicine: A comparison of quality perceptions. Int J Health Care Qual Assur Inc Leadersh Health Serv 11(1):14-20.\r\nAniza, I., Rizal, A.M., Ng, Y.S., Mardhiyyah, M., Helmi, I., Syamimi, B.K. & Tahar, A.R. 2011.Caregivers’ satisfaction of heathcare delivery at paediatric clinics of Universiti Kebangsaan Medical Centre in 2009. Med J Malaysia 66(2):84-88.\r\nAniza, I., Suhaila, A. 2011. Clients satisfactions in ISO certified health clinic in Klinik Kesihatan Bandar Baru Bangi, Selangor and its associated factors. Journal of Community Health 17(1):18-25.\r\nAriff, K.M. & Teng, C.L. 2002. Rural health care in Malaysia. Australia Journal of Rural Health 10(2):99-103.\r\nCoyle, J. & Williams, B. 1999. Seeing the woods for the trees: Defining the forgotten concept of patient dissatisfaction in the light of patient satisfaction research. Int J Health Care Qual Assur Inc Leadersh Health Serv 12(6-7):i-ix\r\nEvans, R.G., Edwards, A., Evans, S., Elwyn, B., & Elwyn, G. 2007. Assessing the practising physician using patient surveys: a systematic review of instruments and feedback methods. Fam Pract 24(2):117-127.\r\nDonabedian, A. 1997. The quality of care: How can it be assessed? Arch Pathol Lab Med 121(11):1145-1150.\r\nDonahue, K.E., Ashkin, E. & Pathman, D.E. 2005. Length of patient-physician relationship and patients’ satisfaction and preventive service use in the rural south: a cross-sectional telephone study. BMC Fam Pract 6:40.\r\nGadallah, M., Zaki, B., Rady, M., Anwer, W. & Sallam, I. 2003. Patient satisfaction with primary health care services in two districts in Lower and Upper Egypt. East Mediterr Health J 9(3):422-430.\r\nGrogan, S., Conner, M., Willits, D., & Norman, P. 1995. Development of a questionnaire to measure patients’ satisfaction with general practitioners’ services. Br J Gen Pract 45:525-529.\r\nHaliza, A.M., Rizal, A.M. & Raja Jamaluddin, R.A.M. 2003. Kajian kepuasan pelanggan di kalangan pesakit klinik swasta di Seremban [A study on customer satisfaction among patients in private clinics in Seremban]. Journal of Community Health 9:44-47.\r\nHaliza, A.M., Rizal, A.M., Raja Jamaluddin, R.A. & Noorhaida, U. 2005. Kepuasan pelanggan di kalangan pesakit luar klinik kerajaan dan klinik swasta di Seremban, N. Sembilan [Customer satisfaction among patients in a government outpatient clinic and private clinics in Seremban, N. Sembilan]. Journal of Community Health 11(1).\r\nHutchinson, A. 1993. The role of patient satisfaction assessment in medical audit. Scand J Prim Health Care Suppl 1:19-22.\r\nIlhaamie, A.G.A. 2010. Service quality in Malaysian public service: Some findings. International Journal of Trade, Economics and Finance 1(1):40-45.\r\nJohn, J., Yatim, F.M., & Mani, S.A. 2011. Measuring service quality of public dental health care facilities in Kelantan, Malaysia. Asia Pac J Public Health 23(5):742-753.\r\nKasalova, H. 1995. Rectification of the primary data obtained by a patients’ satisfaction survey. Int J Health Care QualAssur 8(1):15-16\r\nLai, P.S., Chua, S.S., Chan, S.P., Low, W.Y. & Wong, I.C. 2010. Development and validation of the Osteoporosis Patient Satisfaction Questionnaire (OPSQ). Maturitas 65(1):55-63.\r\nMendoza, A.J., Piechulek, H., al-Sabir, A. 2001. Client satisfaction and quality of health care in rural Bangladesh. Bull World Health Organ 79(6):512-517.\r\nMinistry of Health Malaysia. 2012. Health Facts 2012. Malaysia.\r\nMorgan, E.D., Pasquarella, M., & Holman, J.R. 2004.Continuity of care and patient satisfaction in a family practice clinic. J Am Board Fam Pract 17(5):341-346.\r\nMpinga, E.K. & Chastonay, P. 2011. Patient satisfaction studies and the monitoring of the right to health: Some thoughts based on a review of the literature. Global Journal of Health Science 3(1):64-69.\r\nMohd Suki, N., Chwee Lian, J.L. & Suki, N.M. 2009. A comparison of human elements and nonhuman elements in private health care settings: customers’ perceptions and expectations. J Hosp Mark Public Relations 19(2):113-128.\r\nNoor, S.N. & Musa, S. 2007. Assessment of patients’ level of satisfaction with cleft treatment using the Cleft Evaluation Profile. Cleft Palate Craniofac J 44(3):292-303.\r\nNoor Hazilah, A.M. 2012. Inpatient satisfaction: an analysis of Malaysian public hospitals. International Journal of Public Sector Management 25(1):6-16.\r\nNoor Hazilah, A. & Nooi, P.S. 2009. Patient satisfaction as an indicator of service quality in Malaysian public hospitals. Asian Journal on Quality 10(1):77-87.\r\nNor Hayati, I., Azimatun, N.A., Rozita, H., Sharifa Ezat, W.A. & Rizal, A.M. 2010. In-patients’ satisfaction in the medical and surgical wards -a comparison between accredited and non accreditated hospitals in the state of Selangor. Journal of Community Health 16(1):60-68.\r\nParasuraman, A., Zeithaml, V. & Berry, L. 1985. A conceptual model of service quality and its implications for future research. J Marketing 49(1):41-50\r\nPitaloka, S., Rizal, A. 2006. Patients’ Satisfaction in antenatal clinic Hospital Universiti Kebangsaan Malaysia. Malaysian Journal of Community Health 12(1).\r\nRaja Jamaluddin, R.A.M. 2001. Mengujudkan budaya kualiti di klinik-klinik jagaan kesihatan primer swasta [Establishing a culture of quality in private primary healthcare clinics]. Journal of Community Health 7:19-23.\r\nSaiboon, I., Eng, H.S., Krishnan, B., Ali, S.N., Murad, N., Pathnathan, A. & Choy, C.Y. 2008. A study of patients’ satisfaction with the emergency department (ED) of Hospital UniversitiKebangsaan Malaysia (HUKM). Med & Health 3(1):7-13.\r\nSharifa Ezat, W.P., Jamsiah, M., Malka, S.A., Azimatun Noor, A., Tuti, N., Noor Izzah, A.S. 2010. Customers’ satisfaction among urban and rural public health clinics in state of Selangor, Malaysia. Malaysian Journal of Public Health Medicine 10(2):52-67.\r\nSiow, K.K., Ong, S.T., Lian, C.B. & Ngeow, W.C. 2002. Satisfaction of orthognathic surgical patients in a Malaysian population. J Oral Sci 44(3-4):165-171.\r\nSuki, N.M., Lian, J.C.C. & Suki, N.M. 2011. Do patients’ perception exceed their expectations in private healthcare settings? Int J Health Care Qual Assur 24(1):42-56.\r\nThiedke, C.C. 2007. What do we really know about patient satisfaction? Fam Pract Manag 14(1):33-36.\r\nThung, Y.C. & Chan, G. 2009. Patient satisfaction with and recommendation of a primary care provider: associations of perceived quality and patient education. Int J Qual Health Care 21(3):206-203.\r\nWan Rashid, W.E & Jusoff, H.K. 2009. Service quality in health care setting. Int J Health Care Qual Assur 22(5): 471-482.\r\nWare, J.E. & Snyder, M.K. 1975. Dimensions of patient attitudes regarding doctors and medical care services. Medical Care 13:669–682.\r\nYunus, M.A., Nasir, M.M.T., Nor Afiah, M.Z., Sherina, M.S. & Faizah, M.Z. 2004. Patient satisfaction: A comparison between government and private clinics in Mukim Labu, Sepang, Selangor. Malaysian Journal of Public Health Medicine 4(2):6-11.'),
(933, 928, 'Automated Thyroid Autoantibody Assay', 'Dr. Hanita Othman, Department of Diagnostic Laboratory Services, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaakob Latif, Bandar Tun Razak, Cheras,56000, Kuala Lumpur . Tel: 603-91455449 Fax: 603-91456579. Email: drhanita@ppukm.ukm.edu.my', NULL, 909, 24, 31, 'Bolton, J., Sanders, J., Oda, Y., Chapman, C., Konno, R., Furmaniak, J., Rees Smith, B. 1999. Measurement of thyroid-stimulating hormone receptor autoantibodies by ELISA. Clin Chem 45(12): 2285-2289.\r\nHermsen, D., Broecker-Preuss, M., Casati, M., Mas, J.C., Eckstein, A., Gassner, D., van Helden, J., Inomata, K., Jarausch, J., Kratzsch, J., Mann, K., Miyazaki, N., Navarro Moreno, M.A., Murakami, T., Roth, H.J., Noh, J.Y., Scherbaum, W.A., Schott, M. 2009. Technical evaluation of the first fully automated assay for the detection of TSH receptor autoantibodies. Clin Chem Acta 401(1-2):84-89.\r\nKamijo, K. 2003. TSH-receptor antibody measurement in patients with various thyrotoxicosis and Hashimoto thyroiditis: a comparison of two steps assay using porcine TSH-receptor and human recombinant TSH receptor. Endocr J 50(1):113-116.\r\nKamijo, K. 2007. TSH-receptor antibody determined by first, second and third generation assay. Endocr J 54(4): 619-624.\r\nLiu, C., Hermsen, D., Domberg, J. 2008. Comparison of M22-based ELISA and human-TSH-receptor-based luminescencence assay for the measurement of thyrotropin receptor antibodies in patients with thyroid diseases. Horm Metab Res 40(7):479-483.\r\nMorgenthaler,N.G 1999. New assay systems for thyrotropin receptor antibodies. Curr Opin Endocrinol Diabetes 6:251-260.\r\nSanders, J., Jeffreys, J., Depraetere, H., Richards, T., Evans, M., Kiddie, A., Brereton, K., Groenen, M., Oda, Y., Furmaniak, J., Rees Smith, B. 2002. Thyroid stimulating monoclonal antibodies. Thyroid 12(12):1043-1050.\r\nSaravanan, P., Dayan, C.M. 2001. Thyroid autoantibodies. Endocrinol Metab Clin North Am 30(2):315-337.\r\nSchott, M., Hermsen, D., Martina, B.P., Marco, C.J. 2009. Clinical value of first automated TSH receptor autoantibody assay for the diagnosis of Graves disease: an international multicentre trial. Clin Endocrinol 63 enclosure 6:1-8.\r\nSchott, M., Scherbaum, W.A., Morgenthaler, N.G. 2005. Thyrothropin receptor autoantibodies in Graves disease. Trends Endocrinol Metab 16(5):243-248\r\nSmith, B.R., Bolton, J., Young, S., Collyer, A., Weeden, A., Bradbury, J., Weightman, D., Perros, P., Sanders, J., Furmaniak, J. 2005 A new assay for thyrotropin receptor autoantibodies. Thyroid 14(10):830-835.\r\nSmith, B.R., Sanders, J., Furmaniak, J. 2007. TSH receptor antibodies. Thyroid 17(10):923-38\r\nWeetman, A.P. 2000. Graves’ Disease. N Engl J Med 343(17): 1236- 48.');
INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
(936, 931, 'Downregulation of Bcl-2 by p53 in Breast Cancer', 'Prof. Dr. Sharifah Noor Akmal Syed Husain, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur.Tel: 603-91455372 Fax: 603-91456676 Email: sharifah@ppukm.ukm.edu.my', NULL, 909, 32, 40, 'Abdel-Fatah, T.M., Powe, D.G., Agboola, J., Adamowicz-Brice, M., Blamey, R.W., Lopez-Garcia, M.A., Green, A.R., Reis-Filho, J.S. & Ellis, I.O. 2010. The biological, clinical and prognostic implications of p53 transcriptional pathways in breast cancers. J Pathol 220(4): 419-434.\r\nAllred, D.C., Clark, G.M., Elledge, R., Fuqua, S.A., Brown, R.W., Chamness, G.C., Osborne, C.K. & Mcguire, W.L. 1993. Association of p53 protein expression with tumor cell proliferation rate and clinical outcome in node-negative breast cancer. J Natl Cancer Inst 85(3): 200-206.\r\nBarbareschi, M., Caffo, O., Veronese, S., Leek, R.D., Fina, P., Fox, S., Bonzanini, M., Girlando, S., Morelli, L., Eccher, C., Pezzella, F., Doglioni, C., Dalla Palma, P. & Harris, A. 1996. Bcl-2 and p53 expression in node-negative breast carcinoma: a study with long-term follow-up. Hum Pathol 27(11): 1149-1155.\r\nBartek, J., Bartkova, J., Vojtesek, B., Staskova, Z., Rejthar, A., Kovarik, J. & Lane, D.P. 1990. Patterns of expression of the p53 tumour suppressor in human breast tissues and tumours in situ and in vitro. Int J Cancer. 46(5): 839-844.\r\nBerardo, M.D., Elledge, R.M., De Moor, C., Clark, G.M., Osborne, C.K. & Allred, D.C. 1998. bcl-2 and apoptosis in lymph node positive breast carcinoma. Cancer 82(7): 1296-1302.\r\nBonnefoy-Berard, N., Aouacheria, A., Verschelde, C., Quemeneur, L., Marcais, A. & Marvel, J. 2004. Control of proliferation by Bcl-2 family members. Biochim Biophys Acta. 1644(2-3): 159-168\r\nCharpin, C., Garcia, S., Bonnier, P., Martini, F., Andrac, L., Horschowski, N., Lavaut, M.N. & Allasia, C. 1998. bcl-2 automated and quantitative immunocytochemical assays in breast carcinomas: correlation with 10-year follow-up. J Clin Oncol 16(6): 2025-2031.\r\nColes, C., Condie, A., Chetty, U., Steel, C.M., Evans, H.J. & Prosser, J. 1992. p53 mutations in breast cancer. Cancer Res 52(19): 5291-5298.\r\nCoppola, D., Catalano, E. & Nicosia, S. V. 1999. Significance of p53 and Bcl-2 Protein Expression in Human Breast Ductal Carcinoma. Cancer Control 6(2): 181-187. \r\nDavidoff, A. M., Kerns, B. J., Iglehart, J. D. & Marks, J. R. 1991. Maintenance of p53 alterations throughout breast cancer progression. Cancer Res 51(10): 2605-2610.\r\nGasparini, G., Barbareschi, M., Doglioni, C., Palma, P.D., Mauri, F.A., Boracchi, P., Bevilacqua, P., Caffo, O., Morelli, L., Verderio, P. & Et Al. 1995. 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(1000, 994, 'Parenting Stress in Childhood Leukaemia', 'Prof. Dr. Zarina Abdul Latiff, Department of Paediatrics, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91457888 Fax:+603-91737827 Email: zarinaal@ppukm.ukm.edu.my', NULL, 988, 73, 83, 'Abidin, R.R. 1995. Parenting Stress Index (PSI) Manual. 3rd edn. Charlottesville VA: Pediatric Psychology Press.\r\nEndler, N.S., Parker, J.D.A. 1990. Coping Inventory for Stressful Situations (CISS): Manual. Toronto: Multi-Health Systems.\r\nFarah Inaz, S.A. 2004. Parenting stress in mothers of children with epilepsy. A dissertation for the Masters of Medicine (Pediatrics), Faculty of Medicine Universiti Kebangsaan Malaysia.\r\nFolkman, S., Lazarus, R.S. 1998. The relationship between coping and emotion: Implications for theory and research. Soc Sci Med 26(3): 309-17.\r\nGreaves, M. 2002. Childhood leukaemia. BMJ 324(7332): 283-287.\r\nHoekstra-Weebers, J.E., Jaspers, J.P., Kamps, W.A., Klip, E.D. 1998. Gender differences in psychological adaptation and coping in parents of pediatric cancer patients. Psychooncology 7(1): 26–36.\r\nHoekstra-Weebers, J.E., Jaspers, J.P., Kamps, W.A., Klip, E.C. 2001. Psychological adaptation and social support of parents of pediatric cancer patients: A prospective longitudinal study. J Pediatr Psychol 26(4): 225–235.\r\nKazak, A.E., Boyer, B.A., Brophy, P., Johnson, K., Scher, C.D., Covelman, K., Scott, S. 1995. Parental perceptions of procedure-related distress and family adaptation in childhood leukemia. Child Health Care 24(3): 143–158.\r\nKazak, A.E., Barakat, L.P., Meeske, K., Gallagher, P., Cnaan, A, Stuber, M. 2001. Posttraumatic stress in survivors of childhood cancer and\r\n83\r\nParenting Stress in Childhood Leukaemia Med & Health 2012; 7(2): 73-83\r\ntheir mothers: development and validation of the Impact of Traumatic Stressors Interview Schedule (ITSIS). Journal of Clinical Psychology in Medical Settings 8(4):307–323.\r\nKazak, A.E., Cant, M.C., Jensen, M.M., McSherry, M., Rourke, M.T., Hwang, W.T., Alderfer, M.A., Beele, D., Simms, S., Lange, B.J. 2003. Identifying psychosocial risk indicative of subsequent resource use in families of newly diagnosed pediatric oncology patients. J Clin Oncol 21(17): 3220–3225.\r\nKazak, A.E., Alderfer, M., Rourke, M.T., Simms, S., Streisand, R., Grossman, J.R. 2004. Posttraumatic stress disorder (PTSD) and posttraumatic stress symptoms (PTSS) in families of adolescent childhood cancer survivors. J Pediatr Psychol 29(3): 211-219.\r\nLazarus, R.S., Folkman, S. 1984. Stress, appraisal and coping. New York: Springer Publishing Company 328-333.\r\nLim, G.C.C., Halimah, Y. 2003. The Second Report of the National Cancer Registry - Cancer Incidence in Malaysia. National Cancer Registry\r\nManne, S.L., Du Hamel, K., Gallelli, K., Sorgen, K., Redd, W.H. 1998. Posttraumatic stress disorder among mothers of pediatric cancer survivors: diagnosis, comorbidity, and utility of the PTSD checklist as a screening instrument. J Pediatr Psychol 23(6): 357–366.\r\nMinistry of Health Malaysia. Health Indicator, 2000.\r\nNorshireen Nazli, A.R. 2005. Parental stress in Malaysian mothers of children with spina bifida. Dissertation for the Masters of Medicine (Paediatrics), Faculty of Medicine Universiti Kebangsaan Malaysia.\r\nPearlin, L.I., Schooler, C. 1978. The structure of coping. J Health Soc Behav 19(1): 2-21.\r\nPui, C.H. 2000. Acute lymphoblastic leukemia in children. Curr Opin Oncol 12(1): 3-12.\r\nRay, C., Lindop, J., Gibson, S. 1982. The concept of coping. Psychol Med 12(2): 385-395.\r\nSalwina, W.W.I. 2001. Coping styles, self-concept and stress level among the psychiatry nursing staff and the casualty nursing staff. A dissertation for the Masters of Medicine (Psychiatry), Faculty of Medicine, Universiti Kebangsaan Malaysia.\r\nSawyer, M.G., Antoniou, G., Toogood, I., Rice, M., Baghurst, P.A. 1993. A prospective study of the psychological adjustment of parents and families of children with cancer. J Paediatr Child Health 29(5): 352– 356.\r\nSawyer, M., Antoniou, G., Toogood, I., Rice, M., Baghurst, P. 2000. Childhood cancer: a 4-year prospective study of the psychological adjustment of children and parents. J Pediatr Hematol Oncol 22(3): 214–220.\r\nStreisand, R., Braniecki, S., Tercyak, K.P., Kazak, A.E. 2001. Childhood illness related parenting stress: The pediatric inventory for parents. J Pediatr Psychol 26(3): 155–162.\r\nYeh, C.H. 2002. Gender differences of parental distress in children with cancer. J Adv Nurs 38(6): 598-606.\r\nYoung, B., Dixon-Woods, M., Findlay, M., Heney, D. 2002. Parenting in a crisis: conceptualising mothers of children with cancer. Soc Sci Med 55(10): 1835–1847.'),
(1006, 1000, 'Photoprotective Measures of Sunscreens', 'Dr. Khamsiah Nawawi, Department of Family Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur. Tel: +603 9289 7293 Fax: +603 2693 8037 Email: kham711@gmail.com', NULL, 988, 84, 96, 'Alberts, D.S., Goldman, R., Xu, M.J., Dorr, R.T., Quinn, J., Welch, K., Guillen-Rodriguez, J., Aickin, M., Peng, Y.M., Loescher, L., Gensler, H. 1996. Disposition and metabolism of topically administered alpha-tocopherol acetate: a common ingredient of commercially available sunscreens and cosmetics. Nutr Cancer 26(2): 193-201.\r\nBastuji-Garin, S., Diepgen, T. L., 2002. Cutaneous malignant melanoma, sun exposure and sunscreen use: epidemiological evidence. Br J Dermatol 146(suppl 61):24–30.\r\nCOLIPA Guidelines: In vitro Method for the Determination of the UVA protection factor\r\n96\r\nMed & Health 2012; 7(2): 84-96 Khamsiah N. et al.\r\nand Critical Wavelength Values of Sunscreen Product.COLIPA, p.24.\r\nDiffey, B.L. 2001. When should sunscreen be reapplied?. J Am Acad Dermatol, 45(6), 882-885.\r\nDiffey, B.L., Tanner, P.R., Matts, P.J., Nash, J.F., 2000. In vitro assessment of the broad spectrum ultraviolet protection of sunscreen products. J Am Acad Dermatol 43(6): 1024-1035.\r\nDominique L., 1999. UV Sun conference filters Paris Nov 99, Contribution to measuring In Vitro protection: Some key rules for the reliability of sunscreen spectrophotometric evaluation. http://www.biblioscreen.helioscreen.fr/Documents%20helioscreen/Conference_UV_SUN_%20FILTER_%20Nov99.pdf (Accessed on 14 June 2012)\r\nEC, 2006. European Comission Recommendation on efficacy of sunscreen product and the claims of relating thereto. 2006/647/EC.OJ L 265: 39-43.\r\nEdlich, R.F., Winters, K.L., Lim, H.W., Cox, M.J., Becker, D.G., Horowitz, J.H., Nitcher, L.S., Britt, L.D., Long, W.B. 2004. Photoprotection by Sunscreens with Topical Antioxidants and Systemic Antioxidants to Reduce Sun Exposure. J Long Term Eff Med Implants 14(4): 317–340.\r\nEnvironmental Work Group (EWG) 6th Annual Sunscreen Guide 2012. http://breakingnews.ewg.org/2012sunscreen/ (Accessed on 09 July 2012)\r\nFDA, 2011. Food and Drug Administration. FDA announces new requirements for over-the-counter (OTC) sunscreen products marketed in the U.S. http://www.fda.gov/Drugs/ResourcesForYou/Consumers/BuyingUsingMedicineSafely/UnderstandingOver-the-CounterMedicines/ucm258468.htm (Accessed on 15 July 2012)\r\nGaroli, D., Pelizzo, M.G., Bernardini, B., Nicolosi, P., Alaibac, M. 2008. Sunscreen tests: correspondence between in vitro data and values reported by the manufacturers. J Dermatol Sci 52(3): 193-204.\r\nGilchrest, B.A. 2008. Sun exposure and vitamin D sufficiency. Am J Clin Nutr 88(2): 570S-577S.\r\nGorham, E.D., Mohr, S.B., Garland, C.F., Chaplin, G., Garland, F.C., 2007. Do sunscreen increase risk of melanoma in populations residing at higher latitudes? Ann Epidemiol 17(12): 956-63.\r\nHerzog, B., & Sommer, K. 2000. Investigations on photostability of UV-absorbers for cosmetic sunscreens. In Proceedings (CD-ROM): 21st IFSCC Congress, Berlin; 11-14.\r\nLin, J.Y., Selim, M.A., Shea, C.R., Grichnik, J.M., Omar, M.M., Monteiro-Riviere N.A., Pinnell, S.R. 2003. UV photoprotection by combination topical antioxidants vitamin C and vitamin E. J Am Acad Dermatol 48(6): 866-74.\r\nMurray, J.C., Burch, J.A., Streilein, R.D., Iannacchione, M.A., Hall, R.P., Pinnell, S.R. 2008. A topical antioxidant solution containing vitamins C and E stabilized by ferulic acid provides protection for human skin against damage caused by ultraviolet irradiation. J Am Acad Dermatol 59(3):418-25.\r\nPinnell, S.R., Yang, H., Omar, M., Monteiro-Riviere, N., DeBuys, H.V., Walker, L.C., Wang, Y,, Levine, M. 2001. Topical L-ascorbic acid: percutaneous absorption studies. Dermatol Surg 27(2): 137-42.\r\nSeite, S., Colige, A., Piquemal-Vivenot, P., Montastier, C., Fourtanier, A., Lapière, C., Nusgens, B. 2000. A full-UV spectrum absorbing daily use cream protects human skin against biological changes occurring in photoaging. Photodermatol Photoimmunol Photomed 16(4): 147-55.\r\nWang, S.Q., Osterwalder U., Jung, K. 2011. Ex vivo evaluation of radical sun protection factor in popular sunscreens with antioxidants. J Am Acad Dermatol 65(3):525-30.'),
(1011, 1005, 'Bilateral Retinal Vasculitis', 'Dr. Umi Kalthum Md Noh, Department of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000, Kuala Lumpur. Tel: 603-91455981. Fax: 603-91456733. Email: umi_fizul@yahoo.com', NULL, 988, 97, 101, 'Alvarez, G.G., Roth, V.R., Hodge, W. Ocular tuberculosis: diagnostic and treatment challenges. Int J Infect Dis. 2009;13(4):432-435.\r\nDolin, P.J., Raviglione, M.C., Kochi, A. 1994. Global tuberculosis incidence and mortality during 1990-2000. Bull World Health Organ 72(2): 213-220.\r\nGupta, A., Gupta, V., Arora, S., Dogra, M.R., Bambery, P. 2001. PCR-positive tubercular retinal vasculitis: clinical characteristics and management. Retina 21(5): 435-444.\r\nHelm, C.J., Holland, G.N. 1993. Ocular tuberculosis. Surv Ophthalmol 38(3): 229- 256.\r\nKurup, S.K., Chan, C.C. 2006. Mycobacterium-related ocular inflammatory disease: diagnosis and management. Ann Acad Med Singapore 35(3): 203-209.\r\nNayak, S., Acharjya, B. 2012. Mantoux test and its interpretation. Indian Dermatol Online J 3(1): 2-6.\r\nNeunhöffer, H., Gold, A., Hoerauf, H., Herbort, C., Heiligenhaus, A., Zimmermann, O., Feltgen, N. 2013. Isolated ocular Jarisch-Herxheimer reaction after initiating tuberculostatic therapy in a child. Int Ophthalmol, in press.\r\nNissapatorn, V., Lim, Y.A.L., Jamaiah, I., Man Chin, H., Ilyana, M.Z., Nonaziah, M.Z., Siti Hasifah, A., Kuppusamy, I. Tuberculosis in Malaysia, a continuous surge. Southeast Asian J Trop Med Public Health. 2007; 38: 231- 239\r\nRutherford, M., Alisjahbana, B., Maharani, W., Sampurno, H., van Crevel, R., Hil, P.C. 2010. Sensitivity of the quantiferon-gold in-tube assay in sputum smear positive TB cases in Indonesia. PLoS One 5(8): e12020.\r\nSheu, S.J., Shyu, J.S., Chen, L.M., Chen, Y.Y., Chirn, S.C., Wang, J.S. 2001. Ocular manifestations of tuberculosis. Ophthalmology 108(9): 1580- 1585.\r\nSimsek, H., Alpar, S., Ucar, N., Aksu, F., Ceyhan, I., Gözalan, A., Cesur, S., Ertek, M. 2010. Comparison of tuberculin skin testing and T- SPOT.TB for diagnosis of latent and active tuberculosis. Jpn J Infect Dis 63(2): 99-102.\r\nThompson, M.J., Albert, D.M. 2005. Ocular tuberculosis. Arch Ophthalmol 123(6): 844- 849.'),
(1014, 1008, 'Biliary Ascariasis', 'Sai Sanjeev Murugappa, Female Ward, Hospital Daerah Jelebu, Kuala Klawang, 71600 Jelebu, Negeri Sembilan Darul Khusus. Tel: 06-6139473. Fax: +606 - 6137 531 Email: sanjeevmurugappa@yahoo.com', NULL, 988, 102, 106, 'Adaletl, I., Selcuk, D., Guflien, M., Savafi, C. & Korman, U. 2005. MRCP Findings of biliary ascariasis before and after medical treatment. Turk J Gastroenterol16(2): 98-101\r\nIbrarullah, M.D., Mishra, T., Dash, A.P. & Upadhaya, U.N. 2011. Biliary ascariasis – role of endoscopic intervention. Trop Gastroenterol 32(3): 210-213\r\nMisra, S.P. & Dwivedi, M. 2000. Clinical features and management of biliary ascariasis in a non-endemic area. Postgrad Med J 76(891): 29-32.\r\nOrtega, C.D., Ogawa, N.Y., Rocha, M.S., Blasbalg, R., Motoyama-Caiadao, A.H., Warmbrand, G. & Cerri, G.G. 2010. Helminthic Diseases in Abdomen: An Epidemiologic & Radiologic Overview. Radiographics 30(1): 253-267\r\nOswal, K. & Agarwal, A. 2007. Images: Hepato-biliary ascariasis. Indian Journal of Radiology Imaging, 17(2):77-78\r\nSharma, U.K., Rauniyar, R.K. & Bhatta, N. 2005. Roundworm infestation presenting as acute abdomen in four cases-sonographicdiagnosis. Kathmandu Univ Med J (KUMJ) 3(1-9): 87-90'),
(1020, 1014, 'Ectopic Pregnancy Following LNG-EC', 'Dr. Natasha Ain Mohd Nor, Department of Obstetrics and Gynaecology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000, Kuala Lumpur. Tel: 603-91455949. Fax: 603- 91456672. Email: nat170975@yahoo.com', NULL, 988, 107, 111, 'Camp, S. L., Wilkerson, D.S., & Raine, T. 2003. The benefits and risks of over-the-counter availability of levonorgestrel emergency contraception. Contraception 68(5): 309-317.\r\nCheng, L., Gulmezoglu, A.M., Oel, C.J., Piaggio, G., Ezcurra, E., & Look, P.F. 2004. Interventions for emergency contraception. Cochrane Database Syst Rev (3) CD001324.\r\nCleland , K., Raymond, E., Trussell , J., Cheng , L., & Zhu , H. 2010. Ectopic pregnancy and emergency contraceptive pills: a systemic review. Obstet Gynecol 115(6): 1263-1266.\r\nCroxatto, H.B., Brache, V., Pavez, M., Cochon, L., Forcelledo, M.L., Alvarez, F., Massai, R., Faundes, A. & Salvatierra, A.M. 2004. Pituitary-ovarian function following the standard levonorgestrel emergency contraceptive dose or a single 0.75-mg dose given on the days preceding ovulation. Contraception 70(6): 442-450.\r\nCroxatto, H.B., Ortiz, M.E., & Muller, A.L. 2003. Mechanisms of action of emergency contraception. Steroids 68(10-13): 1095-1098.\r\nGainer, E., Mery, C., & Ulmann, A. 2001. Levonorgestrel-only emergency contraception: real-world tolerance and efficacy. Contraception 64(1): 17-21.\r\nGemzell-Danielsson, K. 2010. Mechanism of action of emergency contraception. Contraception 82(5): 404-409.\r\nGhosh, B., Dadhwal, V., Deka, D., Ramesan, CK., & Mittal, S. 2009. Ectopic pregnancy following levonorgestrel emergency contraception: a case report. Contraception 79(2):155-157.\r\nJohansson, E., Brache, V., Alvarez, F., Faundes, A., Cochon, L., Ranta, S., Lovern, M. & Kumar, N. 2002. Pharmacokinetic study of different dosing regimens of levonorgestrel for emergency contraception in healthy women. Hum Reprod, 17(6): 1472-1476.\r\nKozinszky, Z., Bakken, RT., & Lieng, M. 2011. Ectopic pregnancy after levonorgestrel emergency contraception. Contraception 83(3): 281-283.\r\nMarion, L.L., & Meeks, G.R. 2012. Ectopic pregnancy: history, incidence, epidemiology, and risk factors. Clin Obstet and Gynecol 55(2): 376-386.\r\nMikolajczyk, R.T., & Stanford, J.B. 2007. Levonorgestrel emergency contraception: a joint analysis of effectiveness and mechanism of action. Fertil Steril 88(3), 565-571.\r\nNoe, G., Croxatto, H.B., Salvatierra, A.M., Reyes, V., Villarroel, C., Munoz, C., Morales, G. & Retamales, A. 2011. Contraceptive efficacy of emergency contraception with levonorgestrel given before or after ovulation. Contraception 84(5): 486-492.\r\nPaltieli, Y., Eibschitz, I., Ziskind, G., Ohel, G., Silbermann, M., & Weichselbaum, A. 2000. High progesterone levels and ciliary dysfunction -a possible cause of ectopic pregnancy. J Assist Repro Genet 17(2): 103-106.\r\nShaw, J.L., Dey, S.K., Critchley, H.O., & Horne, A.W. 2010. Current knowledge of the aetiology of human tubal ectopic pregnancy. Hum Reprod Update 16(4): 432-444.\r\nSheffer-Mimouni, G., Pauzner, D., Maslovitch, S., Lessing, B., & Gamzu, R. 2003. Ectopic pregnancies following emergency levonorgestrel contraception. Contraception 67(4): 267-269.\r\nVarma, R., & Gupta, J. 2009. Tubal ectopic pregnancy. Clin Evid (Online) doi:pii: 1406.\r\nWanggren, K., Stavreus-Evers, A., Olsson, C., Andersson, E., & Gemzell-Danielsson, K. 2008. Regulation of muscular contractions in the human fallopian tube through prostaglandins and progestagens. Hum Reprod 23(10), 2359-2368.\r\nXiong, X., Buekens, P., & Wollast, E. 1995. IUD use and the risk of ectopic pregnancy: a meta analysis of case-control studies. Contraception 52(1), 23-34.');
INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
(1026, 1020, 'Acute Megakaryoblastic Leukaemia in a Non-Down Syndrome Child', 'Dr. Nurasyikin Yusof, Haematology Unit, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, Cheras, 56000, Kuala Lumpur. Tel: 0391455373. Fax: 0391737340 Email: drsyikin@ppukm.ukm.edu.my', NULL, 988, 112, 119, 'Athale, U.H., Razzouk, B.I., Raimondi, S.C., Tong, X., Behm, F.G., Head, D.R., Srivastava, D.K., Rubnitz, J.E., Bowman, L., Pui, C.H. & Ribeiro, R.C. 2001. Biology and outcome of childhood acute megakaryoblastic leukaemia: a single institution experience. Blood 97(12): 3727-3732.\r\nBain, B.J. 2007. Diagnosis and classification of acute leukaemia. Postgraduate Haematology. 5th edition. UK:Blackwell Publishing Ltd; 476-490.\r\nBernstein, J., Dastugue, N., Haas, O.A., Harbott, J., Heerema, N.A., Huret, J.L., Landman-Parker, J., LeBeau, M.M., Leonard, C., Mann, G., Pages, M.P., Perot, C., Pirc-Danoewinata, H., Roitzheim, B., Rubin, C.M., Slociak, M. & Viguie, F. 2000. Nineteen cases of the t(1;22)(p13;q13) acute megakaryoblastic leukaemia of infants/children and a review of 39 cases: report from a t(1;22) study group. Leukaemia 14(1): 216-218.\r\nCarroll, A., Civin, C., Schneider, N., Dahl, G., Pappo, A., Bowman, P., Emami, A., Gross, S., Alvarado, C. & Phillips, C. 1991. The t(1;22)(p13;q13) is Nonrandom and Restricted to infants with Acute Megakaryoblastic Leukaemia: A Pediatric Oncology Group Study. Blood 78(3): 748-752.\r\nEdmond, S.K. 2001. Case report: Acute megakaryoblastic leukaemia with complex variant 1;22 translocation. Hong Kong Association of Blood Transfusion and Haematology.\r\nGewirtz, A.M. 1989. Acute megakaryocytic leukaemia. Mayo Clin Proc 64(11): 1447-1451.\r\nLion T, Haas OA, Harbott J, Bannier E, Ritterbach J, Jankovic M, Fink FM, Stojimirovic A, Herrmann J, Riehm HJ, et al. 1992. The translocation t(1;22)(p13;q13) is anonrandom marker specifically associated with acute megakaryocytic leukemia in young children. Blood 79(12): 3325-3330.\r\nMa, Z., Morris, S.W., Valentine, V., Li, M., Herbrick, J.A., Cui, X., Bouman, D., Li, Y., Mehta, P.K., Nizetic, D., Kaneko, Y., Chan, G.C., Chan, L.C., Squire, J., Scherer, S.W. & Hitzler, J.K. 2001. Fusion of two novel genes, RBM15 and MKL1, in the t(1;22)(p13;q13) of acute megakaryoblastic leukemia. Nat Genet 28(3): 220-221.\r\nMercher, T., Raffel, G.D., Moore, S.A., Cornejo, M.G., Baudry-Bluteau, D., Cagnard, N., Jesneck, J.L., Pikman, Y., Cullen, D., Williams, I.R., Akashi, K., Shigematsu, H., Bourquin, J.P., Giovannini, M., Vainchenker, W., Levine, R.L., Lee, B.H., Bernard, O.A. & Gilliland, D.G. 2009 The OTT-MAL fusion oncogene activates RBPJ-mediated transcription and induces acute megakaryoblastic leukaemia in a knockin mouse model. The Journal of Clinical Investigation 119(4): 852-864.\r\nRavindranath, Y., Abella, E., Krischer, J.P., Wiley, J., Inoue, S., Harris, M., Chauvenet, A., Alvarado, C.S., Dubowy, R., Ritchey, A.K., et al. 1992. Acute myeloid leukaemia in Down syndrom is highly responsive to chemotherapy: experience on Pediatric Oncology Group AML study 8498. Blood 80(9): 2210-2214.\r\nReinhardt, D., Diekamp, S., Langebrake, C., Ritter, J., Dworzak, M., Schrauder, A., Zimmermann, M., Fleischhack, G., Ludwig, W.D., Harbott, J. & Creutzig, U. 2005. Acute Megakaryoblastic Leukaemia in children and adolescents, excluding Down syndrome: improved outcome with intensified induction treatment. Leukaemia 19(8): 1495-1496.\r\nRibeiro, R.C., Oliveira, M.S., Fairclough, D., Hurwitz, C., Mirro, J., Behm, F.G., Head, D., Silva, M.L., Raimondi, S.C. & Crist, W.M. 1993. Acute megakayoblastic leukaemia in children and adolescent : a retrospective analysis of 24 cases. Lymphoma 10(4-5): 299-306.\r\nSwerdlow, S.H., Campo, E., Harris, N.L., et al. 2008. WHO Classification of Tumours of Haematopoeitic and Lymphoid tissues. 4th edition; 17, 117, 136-137.\r\nVerschuur, A.C. 2004. Acute megakaryoblastic leukaemia. Orphanet Encyclopedia; 1-4 http://www.orpha.net/data/patho/GB/uk-AWLM7.pdf. [20 December 2011]'),
(1040, 1033, 'Palmaris Longus Muscle: Presence or Absence, Does It Matter?', 'Prof. Dr. Farihah Suhaimi, Department of Anatomy, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abd Aziz, 50300 Kuala Lumpur, Malaysia Tel: 06-03-92897219 Fax: 06-03-26989506 Email: farihah@medic.ukm.my', NULL, 1030, 1, 2, 'Cetin, A., Genc, M., Sevil, S., Coban, Y.K. 2013. Prevalence of the palmaris longus muscle and its relationship with grip and pinch strength: a study in a Turkish pediatric population. Hand (NY) 8(2): 215-220.\r\nMurabit. A., Gnarra, M., Mohamed, A. 2013. Reversed palmaris longus muscle: Anatomical variant - case report and literature review. Can J Plast Surg 21(1): 55-56.\r\nMoore, K.L., Dalley, A.F., Agur, A.M.R. 2010. Clinically Oriented Anatomy. 6th ed. Baltimore:Wolters Kluwer/ Lippincott Williams and Wilkins; 748.\r\nRaouf, H.A., Kader, G.A., Jaradat, A., Dharap, A., Fadel, R., Salem, A.H. 2013. Frequency of palmaris longus absence and its association with other anatomical variations in the Egyptian population. Clin Anat 26(5): 572-577.\r\nSoldado-Carrera, F., Vilar-Coromina, N., Rodríguez-Baeza, A. 2000. An accessory belly of the abductor digiti minimi muscle: a case report and embryologic aspects. Surg Radiol Anat 22(1): 51-54.\r\nSoltani, A.M., Peric, M., Francis, C.S., Nguyen, T.T., Chan, L.S., Ghiassi, A., Stevanovic, M.V., Wong, A.K. 2012. The variation in the absence of the palmaris longus in a multiethnic population of the United States: an epidemiological study. Plast Surg Int ID282959.\r\nStack, H.G. 1973. The Palmar Fascia. Edinburgh: Churchill Livingstone; 27-37\r\nWilliams PL. 1995. Gray’s Anatomy: The Anatomical Basis of Medicine and Surgery. 38th edn, 1995, New York: Elsevier/Churchill Livingstone; 846.'),
(1042, 1035, 'Honey: Food or Medicine?', 'Prof. Dr. Yasmin Anum Mohd Yusof, Department of Biochemistry, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Raja Muda Abdul Aziz, 50300 Kuala Lumpur, Malaysia. Tel: +603-92897297 Fax: +603-26938037 Email: anum@medic.ukm.my/rahmatyasmin@yahoo.com', NULL, 1030, 3, 18, 'Abdelhafiz, A.T., Muhamad, J.A. 2008. Midcycle pericoital intravaginal bee honey and royal jelly for male factor infertility. Int J Gynaecol Obstet 101(2): 146-149.\r\nAbdul-Ghani, A.S., Dabdoub, N., Muhammad, R., Abdul-Ghani, R., Qazzaz, M. 2008. Effect of Palestinian honey on spermatogenesis in rats. J Med Food 11(4): 799-802.\r\nAbdul Kadir, E., Sulaiman, S.A., Yahya, N.K., Othman, N.H. 2013. Inhibitory Effects of Tualang Honey on Experimental Breast Cancer in Rats: A Premilinary Study. Asian Pac J Cancer Prev 14(4):2249-2254.\r\nAdebolu, T.T. 2005. Effect of natural honey on local isolates of diarrhea causing bacteria in South Western Nigeria. Afri J Biotech 4(10): 1172-1174.\r\nAgbaje, E.O., Ogunsanya, T., Aiwerioba, O.I.R. 2006. Conventional use of honey as antibacterial agent. Annals Afri Med 5(2): 78-81.\r\nAjlouni, S., Sujirapinyokul, P. 2010. Hydroxymethylfurfuraldehyde and amylase contents in Australian honey. Food Chem 119: 1000-1005.\r\nAl-Jabri, A.A., Al-Hosni, S.A., Nzeako, B.C., Al-Mahrooqi, Z.H., Nsanze, H. 2005. Antibacterial activityof Omani honey alone and in combination with gentamicin. Saudi Med J 26(5): 767-771.\r\nAljadi, A.M., Kamaruddin, M.Y., Jamal, A.M., Mohd Yassim, M.Y. 2000. Biochemical study on the efficacy of Malaysian honey on inflicted wounds: An aminal model. Medical Journal of Islamic Academy of Sciences 13(3): 125-132.\r\nAllen, K.L., Molan, P.C., Reid, G.M. 1991. A survey of the antibacterial activity of some New Zealand honeys. J Pharm Pharmacol 43(12): 817-822.\r\nAllen, K.L., Hutchinson, G., Molan, P.C. 2000. The potential for using honey to treat wounds infected with MRSA and VRE: 10-13 September 2000: First World Healing Congress, Melbourne, Australia.\r\nAl-Mamary, M., Al-Meeri, A., Al-Habori, M. 2002. Antioxidant activities and total phenolics of different types of honey. Nutr Res 22(9): 1041-1047.\r\nAl-Qassemi, R.A.S., Robinson, R.K. 2003. Some special nutritional properties of honey-a brief review. Nutr Fd Sci 33(6): 254-260.\r\nAl-Qur’an. Surah Al-Nahl (The bee) 16: 68-69. http://www.quran.com/16/68-69. (18 August 2012).\r\nAl-Qur’an. Surah Muhammad 47: 15. http://www.quran.com/47/15. (18 August 2102).\r\nAlvarez-Suarez, J.M., Tulipani, S., Romandini, S., Bertoli, E., Battino, M. 2010. Contribution of honey in nutrition and human health: a review. Mediterranean Journal of Nutrition and Metabolism 3: 15-23.\r\nAl-Waili, N.S. 2003. Effects of daily consumption of honey solution on hematological indices and food levels of minerals and enzymes in normal individuals. J Med Food 6(2): 135-40.\r\nAsiyah, H.A., Syazana, N.S., Hashida, N.H., Durriyyah Sharifah, H.A., Kamaruddin, M.Y. 2011. Effects of nicotine and Gelam honey on testis parameters and sperm qualities of juvenile rats. Sci Res Essays 6(26): 5471-5474.\r\nBang, L.M., Buntting, C., Molan, P.C. 2003. The effect of dilution on the rate of hydrogen peroxide production in honey and its implications for wound healing. J Altern Complement Med 9(2): 267-73.\r\nBansal, V., Medhi, B., Pandhi, P. 2005. Honey a remedy rediscovered and its therapeutic utility. Kathmandu Univ Med J (KUMJ) 3(3): 305-9.\r\nBenefits of Honey. 2012. http://www.benefits-of-honey.com/health-benefits-of-honey.html. (11 September 2012).\r\nBlair, S.E., Carter, D. 2005. The potential for honey in the management of wounds and infections. J Australian Infection Control 10(1): 24-31.\r\nBogdanov, S., Haldimann, M., Luginbuhl, W., Gallmann, P. 2007. Minerals in honey: environment, geographical and botanical aspects. Journal of Apicultural Research Bee World 46(4): 269-275.\r\nBogdanov, S., Jurendic, T., Sieber, R., Gallmann, P. 2008. Honey for nutrition and health: a review. J Am Coll Nutr 27(6): 677-89.\r\nBogdanov, S. 2009a. Honey as Nutrient and Functional Food: A Review. http://www.apitherapie.ch/files/files/Honig/8HoneyNutrientFunctionalReview. (26 November 2011).\r\nBogdanov, S. 2009b. Honey Composition. The Honey Book, Chapter 5. http://www.fantasticflavour.com/yahoo_site_admin/assets/docs/CompositionHoney20105942. (20 January 2012).\r\nBogdanov, S. 2010a. Honey in Medicine. The Honey Book. http://www.bee-hexagon.net/files/file/fileE/HealthHoney/9HoneyMedicineReview. (20 October 2011).\r\nBogdanov, S. 2010b. Elaboration and harvest of honey. The Honey Book, Chapter 8. http://www.bee-hexagon.net/files/file/fileE/Honey/2HoneyElaboration. (12 February 2012).\r\nCodex Alimentarius Commission. 2001. Revised Codex standard for honey. http://www.bee-hexagon.net/files/file/fileE/ IHCPapers/Codex2001. [12 December 2011].\r\nCotte, J.F., Casabianca, H., Chardon, S., Lheritier, J., Grenier-Loustalot, M.F. 2004. Chromatographic analysis of sugars applied to the characterisation of monofloral honey. Anal Bioanal Chem 380(4): 698-705.\r\nCrane, E. 1975. History of honey. In Crane E (ed.): Honey, a comprehensive survey. William Heinemann, London; 439-488.\r\nCuevas-Glory, L.F., Pino, J.A., Santiago, L.S., Sauri-Duch, E. 2007. A review of volatile analytical methods for determining the botanical origin of honey. Food Chemistry 103: 1032-1043.\r\nErejuwa, O.O., Sulaiman, S.A., Wahab, M.S., Sirajudeen, K.N., Salleh, M.S., Gurtu, S. 2010. Antioxidant protection of Malaysian Tualang honey in pancreas of normal and streptozotocin-induced diabetic rats. Ann Endocrinol (Paris) 71(4): 291-6.\r\nErejuwa, O.O., Sulaiman, S.A., Wahab, M.S.A., Sirajudeen, K.N.S., Salleh, S., Gurtu, S. 2012. Honey supplementation in spontaneously hypertensive rats elicits antihypertensive effect via amelioration of renal oxidative stress. Oxid Med Cell Longev ID374037\r\nEnglish, H.K., Pack, A.R., Molan, P.C. 2004. The effects of Manuka honey on plaque and gingivitis: a polit study. J Int Acad Periodontol 6(2): 63-7.\r\nFauzi, A.N., Norazmi, M.N., Yaacob, N.S. 2011. Tualang honey induces apoptosis and disrupts the mitochondrial membrane potential of human breast and cervical cancer cell lines. Food Chem Toxicol 49(4): 871-8.\r\nFerreira I. C. F. R., Aires E., Barreira J. C. M., Estevinho L. M. 2009. Antioxidant activity of Portuguese honey sample: Different contributions of the entire honey and phenolics extract. Food Chem 114(4): 1438-1443\r\nFinola, M.S., Lasagno, M.C., Marioli, J.M. 2007. Microbiological and chemical characterization of honeys from central Argentina. Food Chem 100: 1649-1653.\r\nGharzouli, K., Amira, S., Gharzouli, A., Khennouf, S. 2002. Gastroprotective effects of honey and glucose-fructose-sucrose-maltose mixture against ethanol-, indomethacin-, and acidified aspirininduced lesions in the rat. Exp Toxicol Pathol 54(3): 217-221.\r\nGhashm, A.A., Othman, N.H., Khattak, M.N., Ismail, N.N., Saini, R. 2010. Antiproliferative effect of Tualang honey on oral squamous cell carcinoma and osteosarcoma cell lines. BMC Complement Altern Med 10: 49.\r\nGoldstein, S.L., Leung, J.C., Silverstein, D.M. 2006. Pro- and anti-Inflammatory cytokines in chronic pediatric dialysis patients: effect of aspirin. Clin J Am Soc Nephrol 1(5): 979-986.\r\nHamouda, H.M., Abouwarda, A. 2011. Antimicrobial activity of bacterial isolates from honey. International Journal of Microbiological Research 2(1): 82-85.\r\nHanada, T., Yoshimura, A. 2002. Regulation of cytokine signaling and inflammation. Cytokine Growth Factor Rev 13(4-5): 413-421.\r\nTran, M.H., Nguyen, H.D., Kim, J.C., Choi, J.S., Lee, H.K., Min, B.S. 2009. 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(1048, 1041, 'Left Ventricular Stab Wound Embolus', 'Dr. Nik Azlan Nik Muhamad, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 03-91455703 Fax: 03-91456577 Email: nikazlanmuhamad@hotmail.com', NULL, 1030, 28, 32, 'Kertes, P., Westlake, G., Luxton, M. 1983. Multiple Peripheral Emboli after Cardiac Trauma. Br Heart J 49(2): 187-189.\r\nNeidlinger, N.A., Puzziferri, N., Victorino, G.P., Ursic, C.M. 2004. Cardiac Thromboemboli Complicating A Stab Wound to the Heart. Cardiovasc Pathol 13(1): 56-58.\r\nSharma, N.D., McCullough, P.A., Philbin, E.F., Weaver, W.D. 2000. Left Ventricular Thrombus and Subsequent Thromboembolism in Patients With Severe Systolic Dysfunction. Chest 117(2): 314-320.\r\nTayal, V.S., Beatty, M.A., Marx, J.A., Tomaszewski, C.A., Thomason, M.H. 2004. FAST (focused assessment with sonography in trauma) accurate for cardiac and intraperitoneal injury in penetrating chest trauma. J Ultrasound Med 23(4): 467–472.'),
(1053, 1046, 'Epidural Analgesia on Pain Management', 'Assoc. Prof. Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Center, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 603-91456262 Fax: 603-91456683 Email: ho@ppukm.ukm.my/ angieho88@gmail.com', NULL, 1030, 33, 36, 'Capogna, G., Camorcia, M., Stirparo, S. 2007. Expectant father’s experience during labor with or without epidural analgesia. Int J Obstet Anesth 16(2): 110-115.\r\nCatterall, W.A., Mackie, K. 2006. Local anesthetics. In: Brunton, L.L., Lazo, J.S., Parker, K.L. (eds). Goodman & Gilman’s the pharmacological basis of therapeutics. 11th edn. New York: McGraw-Hill, Medical Publishing Division; 369-386.\r\nDickinson, J.E., Paech, M.J., McDonald, S.J., Evans, S.F. 2003. Maternal satisfaction with childbirth and intrapartum analgesia in nulliparous labor. Aust N Z J Obstet Gynaecol 43(6): 463-468.\r\nHansen, S.L., Clark, S.L., Foster, J.C. 2002. Active pushing versus passive fetal descent in the second stage of labor: a randomized controlled trial. Obstet Gynecol 99(1):29-34.\r\nHiltunen, P., Raudaskoski, T., Ebeling, H., Moilanen, I. 2004. Does pain relief during delivery decrease the risk of postnatal depression? Acta Obstet Gynecol Scand 83(3): 257-261.\r\nKumar, M., Paes, B. 2003. Epidural opioid analgesia and neonatal respiratory depression. J Perinatol 23(5):425-427.\r\nNiesen, A.D., Jacob, A.K. 2013. Combined spinal-epidural versus epidural analgesia for labor and delivery. Clin Perinatol 40(3):373-384.\r\nPugliese, P.L., Cinnella, G., Raimondo, P., De Capraris, A., Salatto, P., Sforza, D., Menga, R., D’Ambrosio, A., Fede, R.N., D’Onofrio, C., Consoletti, L., Malvasi, A., Brizzi, A., Dambrosio, M. 2013. Implementation of epidural analgesia for labor: is the standard of effective analgesia reachable in all women? An audit of two years. Eur Rev Med Pharmacol Sci 17(9): 1262-1268.\r\nSociety of Obstetricians and Gynecologists (SOGC). 2004. Cesarean Section on Demand – SOGC’s position. Ottawa: Society of Obstetricians and Gynaecologists of Canada. http://apps.fims.uwo.ca/NewMedia2007/resources/10/reports/Elective_Caesareans_-_position_statement-2004[1].pdf. [Accsessed on 17 January 2014]'),
(1056, 1049, 'Homemade Remedies Causing Dermatitis', 'Assoc. Prof. Dr. Leelavathi Muthupalaniappen, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Center, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 603 91456123 Fax: 603 9145 6688 Email: drleelaraj@gmail.com', NULL, 1030, 37, 41, 'Arthur, B.K. 1960. Contact dermatitis from cinnamon.Arch Dermatol 81(4):599-600.\r\nDaud, F.S., Pande, G., Joshi, M., Pathak, R., Wankhede, S. 2013. Study of Antibacterial Effect of Some Selected Essential Oils and Medicinal Herbs against Acne Causing Bacteria. International Journal of Pharmaceutical Science Invention 2(1): 27-34.\r\nDuus Johansen, J. 2002. Contact allergy to fragrances: clinical and experimental investigations of the fragrance mix and its ingredients. Contact Dermatitis 46 (Suppl 3):4–31.\r\nKimber, I., Basketter, D.A., Gerberick, G.F., Dearman, R.J. 2002. Allergic contact dermatitis. Int Immunopharmacol 2(2-3):201–211.\r\nMatura, M., Goossens, A., Bordalo, O., Garcia-Bravo, B., Magnusson, K., Wrangsjö, K., Karlberg, A.T. 2002. Oxidized citrus oil (Rlimonene): a frequent skin sensitizer in Europe. J Am Acad Dermatol 47(5):709–714.\r\nMagin PJ, Adams J, Heading GS, Pond DC, Smith W. 2006. Complementary and alternative medicine therapies in acne, psoriasis, and atopic eczema: results of a qualitative study of patients’ experiences and perceptions. J Altern Complement Med 12(5):451-457.\r\nRobert, L.R., Joseph, F.F., Alexander, A. F. 2008. In Fisher’s Contact Dermatitis. 6th edition. Hamilton: BC Decker Inc., 456.\r\nSpiewak, R. 2008. Patch testing for contact allergy and allergic contact dermatitis. The Open Allergy Journal 1:42-51.\r\nThomson, M.A., Preston, P.W., Prais, L., Foulds, I.S. 2007. Lime dermatitis from gin and tonic with a twist of lime. Contact Dermatitis 56(2):114–115\r\nTremblay, S., Avon, S.L. 2008. Contact allergy to cinnamon: case report. J Can Dent Assoc 74(5):445-461.\r\nvan den Akker, T.W., Roesyanto-Mahadi, I.D., van Toorenenbergen, A.W., van Joost, T.1990. Contact allergy to spices. Contact Dermatitis 22(5):267-272.'),
(1058, 1051, 'Emergency Department Overcrowding (EDOC)', 'Dr. Nik Azlan Nik Muhamad, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latiff, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 03-91455703 Fax: 03-91456577 Email: nikazlanmuhamad@hotmail.com', NULL, 1030, 42, 46, 'Affleck, A., Innes, G. 2003. Quality of care in walk-in clinics, family practice and emergency departments: the Ontario walk-in clinic study. CJEM 5(5): 350-352.\r\nBond, K., Ospina, M.B., Blitz, S., Afilalo, M., Campbell, S.G., Bullard, M., Innes, G., Holroyd, B., Curry, G., Schull, M., Rowe, B.H. 2007. Frequency, determinants and impact of overcrowding in emergency departments in Canada: a national survey. Healthc Q 10(4): 32-40.\r\nEmergency Department. 2011. Annual Report. Universiti Kebangsaan Malaysia Medical Centre.\r\nForero R, McCarthy S, Hillman K. 2011. Access block and emergency department overcrowding. Crit Care 15(2):216.\r\nGraff, L. 1999. Overcrowding in the ED: an international symptom of health care system failure. Am J Emerg Med 17(2): 208-209.\r\nHutchison, B., Ostbye, T., Barnsley, J., Stewart, M., Mathews, M., Campbell, M.K., Vayda, E., Harris, S.B., Torrance-Rynard, V., Tyrrell, C. 2003. Patient satisfaction and quality of care in walk-in clinics, family practices and emergency departments: the Ontario walk-in clinic study. CMAJ 168(8): 977-983.\r\nKollek, D. 1990. Overwhelmed in emergency: examining ER status at Hamiliton Civic Hospitals. Ont Med Rev 57: 11-13.\r\nLynn, S.G., Kellermann, A.L. 1991. Critical decision making: managing the emergency department in an overcrowded hospital. Ann Emerg Med 20(3): 287-292.\r\nSchull, M.J., Slaughter, P.M., Redelmeier, D.A. 2002. Urban emergency department overcrowding: defining the problem and eliminating misconceptions. CJEM 4(2): 76-83.\r\nYoon, P. 2003. Emergency department fast-track system. HTA Initiative #10. Edmonton: Alberta Heritage Foundation for Medical Research; 11.'),
(1060, 1053, 'Masquerade Syndrome: An Ocular Involvement', 'Dr. Umi Kalthum Md Noh, Department of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Center, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 603-91455981 Fax: 603- 91456673 Email: umi_fizul@yahoo.com', NULL, 1030, 47, 51, 'Gass, J.D., Little, H. 1995. Bilateral bullous exudative retinal detachment complicating idiopathic central serous chorioretinopathy during systemic corticosteroid therapy. Ophthalmology 102(5): 737-747.\r\nKivella, T., Eskelin, S., Makitie, T., Summanen, P. 2001. Exudative retinal detachment from malignant uveal melanoma: predictors and prognostic significance. Invest Ophthalmol Vis Sci 42(9): 2085-2093.\r\nKreusel, K.M., Wiegel, T., Stange, M., Bornfeld, N., Hinkelbein, W., Foerster, M.H. 2002. Choroidal metastasis in disseminated lung cancer: frequency and risk factors. Am J Ophthalmol 134(3): 445- 447.\r\nRosset, A., Zografos, L., Coucke, P., Monney, M., Mirimanoff, R.O. 1998. Radiotherapy of choroidal metastases. Radiother Oncol 46(3): 263- 238.\r\nSkarin, A. 1999. Diagnosis in oncology: choroidal metastases. J Clin Oncol 17(10): 3356- 9.\r\nTsina, E.K., Lane, A.M., Zacks, D.N., Munzenrider, J.E., Collier, J.M., Gragoudas, E.S. 2005. Treatment of metastatic tumors of the choroid with proton beam irradiation. Ophthalmology 112(2): 337- 343.\r\nYang, P., Cerhan, J.R., Vierkant, R.A., Olson, J.E., Vachon, C.M., Limburg, P.J., Parker, A.S., Anderson, K.E., Sellers, T.A. 2002. Adenocarcinoma of the lung is strongly associated with cigarette smoking: Further evidence from prospective study of women. Am J Epidemiol 156(12): 1114- 1122.\r\nZamiri, P., Boyd, S., Lightman, S. 1997. Uveitis in the elderly- is it easy to identify the masquerade? Br J Ophthalmol 81(10): 827- 831.'),
(1098, 1090, 'Is Bariatic Surgery The Cure for Diabetes Mellitus?', 'Dr. Nik Ritza Kosai Nik Mahmood, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia Tel: +603-91456201 Fax: +603-91456684 Email: nikkosai@yahoo.co.uk', NULL, 1088, 52, 54, 'Chan, J.M., Stampfer, M.J., Ribb, E.B. 1994. Obesity, fat distribution, and weight gain as risk factors for clinical diabetes in men. Diabetes Care 17: 961-9.\r\nBuchwald, H., Estok, R., Fahrbach, K., Banel, D., Jensen, M.D., Pories, W.J., Bantle, J.P., Sledge, I. 2009. Weight and type 2 diabetes after bariatric surgery: systematic review and meta-analysis. Am J Med 122(3): 248-56.\r\nFriedman, M.N., Sancetta, A.J., Magovern, G.J. 1955. The amelioration of diabetes mellitus following subtotal gastrectomy. Surg Gynecol Obstet 100(2): 201-4.\r\nKlein, S., Ghosh, A., Cremieux, P.Y., Eapen, S., McGavock, T.J. 2011. Economic impact of the clinical benefits of bariatric surgery in diabetes patients with BMI≥35 kg/m. Obesity (Silver Spring) 19(3): 581-7.\r\nPapamargaritis, D., Miras, A.D., le Roux, C.W. 2013. Influence of diabetes surgery on gut hormones and incretins. Nutr Hosp 28(Suppl 2): 95-103.\r\nPatriti, A., Facchiano, E., Annetti, C., Aisa, M.C., Galli, F., Fanelli, C., Donini, A. 2005. Early improvement of glucose tolerance after ileal transportation in a non-obese type 2 diabetes rat model. Obes Surg 15(9): 1258-64.\r\nRubino, F., Forgione, A., Cummings, D.E., Vix, M., Gnuli, D., Mingrone, G., Castagneto, M., Marescaux, J. 2006. The mechanism of diabetes control after gastrointestinal bypass surgery reveals a role of the proximal small intestine in the pathophysiology of type 2 diabetes. Ann Surg 244(5): 741-9.\r\nSchauer, P.R., Bhatt D.L., Kirwan, J.P., Wolski, K., Brethauer, S.A., Navaneethan, S.D., Aminian, A., Pothier, C.E., Kim, E.S.H., Nissen, S.E., Kashyap S.R. 2014. Bariatric surgery versus intensive medical therapy for diabetes-3 year outcomes. N Engl J Med 370: 2002-13.\r\nSjostrom, L. 2013. Review of the key results from Swedish obese subjects (SOS) trial-a prospective controlled intervention study of Bariatric Surgery. J Intern Med 273(3): 219-34.');
INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
(1105, 1097, 'CKD Stage 3B in Diabetics', 'Dr. Noorlaili Mohd Tauhid, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456117 Fax: +603-91456680 Email: lailitauhid@yahoo.com', NULL, 1088, 55, 63, 'Bakris, G., Weir, M.R., Shanifar, S., Zhang, Z., Douglas, J., van Dijik D.J., Brenner B.M. 2003. Effects of blood pressure level on progression of diabetic nephropathy: results from the RENAAL study. Arch Intern Med 163(13): 1555-5.\r\nCenters for Disease Control and Prevention (CDC). 2007. Prevalence of chronic kidney disease and associated risk factors--United States, 1999-2004. MMWR Morb Mortal Wkly Rep 56(8): 161-5.\r\nChan, G.C. 2005. Type 2 diabetes mellitus with hypertension at primary healthcare level in Malaysia: are they managed according to guidelines? Singapore Med J 46(3): 127-31.\r\nChen, N., Wang, W., Huang, Y., Shen, P., Pei, D., Yu, H., Shi, H., Zhang, Q., Xu, J., Lv, Y., Fan, Q. 2009. Community-based study on CKD subjects and the associated risk factors. Nephrol Dial Transplant 24(7): 2117-23.\r\nCheong, A.T., Tong, S.F., Sazlina, S.G., Azah, A.S., Salmiah, M.S. 2013. Blood Pressure Control Among Hypertensive Patients With and Without Diabetes Mellitus in Six Public Primary Care Clinics in Malaysia. Asia Pac J Public Health doi: 10.1177/1010539513480232.\r\nChew, B.H., Mastura, I., Cheong, A.T., Syed Alwi, S.A.R. 2010. Diabetic hypertensive control and treatment: a descriptive report from the Audit Diabetes Control and Management (ADCM) Registry. Malaysian Family Physician 5(3): 134-9.\r\nChia, Y.C., Ching, S.M. 2012. Hypertension and the development of new onset chronic kidney disease over a 10 year period: a retrospective cohort study in a primary care setting in Malaysia. BMC Nephrol 13: 173.\r\nCockcroft, D.W., Gault, M.H. 1976. Prediction of creatinine clearance from serum creatinine. Nephron 16(1): 31-41.\r\nHaroun, M.K., Jaar, B.G., Hoffman, S.C., Comstock, G.W., Klag, M.J., Coresh, J. 2003. Risk factors for chronic kidney disease: a prospective study of 23,534 men and women in Washington County, Maryland. J Am Soc Nephrol 14(11): 2934-41.\r\nHeart Outcomes Prevention Evaluation Study Investigators. 2000. Effects of ramipril on cardiovascular and microvascular outcomes in people with diabetes mellitus: results of the HOPE study and MICRO-HOPE substudy. Lancet 355(9200): 253-9.\r\nHooi, L.S., Ong, L.M., Ahmad, G., Bavanandan, S., Ahmad, N.A., Naidu, B.M., Mohamud, W.N., Yusoff, M.F. 2013. A population-based study measuring the prevalence of chronic kidney disease among adults in West Malaysia. Kidney Int 84(5): 1034-40.\r\nHsu, C.Y., Lin, F., Vittinghoff, E., Shlipak, M.G. 2003. Racial differences in the progression from chronic renal insufficiency to end-stage renal disease in the United States. J Am Soc Nephrol 14(11): 2902-7.\r\nIngsathit, A., Thakkinstian, A., Chaiprasert, A., Sangthawan, P., Gojaseni, P., Kiattisunthorn, K., Ongaiyooth, L., Vanavanan, S., Sirivongs, D., Thirakhupt, P., Mittal, B., Singh, A.K. 2010. Prevalence and risk factors of chronic kidney disease in the Thai adult population: Thai SEEK study. Nephrol Dial Transplant 25(5): 1567-75.\r\nKeane, W.F., Brenner, B.M., de Zeeuw, D., Grunfeld, J.P., McGill, J., Mitch, W.E., Ribeiro, A.B., Shahinfar, S., Simpson, R.L., Snapinn, S.M., Toto, R. 2003. The risk of developing end-stage renal disease in patients with type 2 diabetes and nephropathy: the RENAAL study. Kidney Int 63(4): 1499-1507.\r\nLevey, A.S., Coresh, J., Balk, E., Kausz, A.T., Levin, A., Steffes, M.W., Hogg, R.J., Perrone, R.D., Lau, J., Eknoyan, G., 2003. National Kidney Foundation practice guidelines for chronic kidney disease: evaluation, classification, and stratification. Ann Intern Med 139(2): 137-47.\r\nLevey, A.S., de Jong, P.E., Coresh, J., El Nahas, M., Astor, B.C., Matsushita, K., Gansevoort, R.T., Kasiske, B.L., Eckardt, K.U. 2011. The definition, classification, and prognosis of chronic kidney disease: a KDIGO Controversies Conference report. Kidney Int 80(1): 17-28.\r\nLi, L., Astor, B.C., Lewis, J., Hu, B., Appel, L.J., Lipkowitz, M.S., Toto, R.D., Wang, X., Wright, J.T., Greene, T.H. 2012. Longitudinal progression trajectory of GFR among patients with CKD. Am J Kidney Dis 59(4): 504-12.\r\nMeguro, S., Shigihara, T., Kabeya, Y., Tomita, M., Atsumi, Y. 2009. Increased risk of renal deterioration associated with low e-GFR in type 2 diabetes mellitus only in albuminuric subjects. Intern Med 48(9): 657-63.\r\nNational Kidney Foundation. 2004. Use of Angiotensin-Converting Enzyme Receptor Blockers in CKD. K/DOQI Clinical Practice Guidelines on Hypertension and Antihypertensive Agents in Chronic Kidney Disease Guideline 11. New York.\r\nNational Renal Registry. 2011. 18th Report of the Malaysian Dialysis & Transplant Registry 2010. Kuala Lumpur.\r\nOng-Ajyooth, L., Vareesangthip, K., Khonputsa, P., Aekplakorn, W. 2009. Prevalence of chronic kidney disease in Thai adults: a national health survey. BMC Nephrol 10: 35.\r\nOrth, S.R., Hallan, S.I. 2008. Smoking: a risk factor for progression of chronic kidney disease and for cardiovascular morbidity and mortality in renal patients--absence of evidence or evidence of absence? Clin J Am Soc Nephrol 3(1): 226-36.\r\nRavid, M., Brosh, D., Levi, Z., Bar-Dayan, Y., Ravid, D., Rachmani, R. 1998. Use of enalapril to attenuate decline in renal function in normotensive, normoalbuminuric patients with type 2 diabetes mellitus. A randomized, controlled trial. 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(1109, 1101, 'Clostridium difficile Infection', 'Dr Ramliza Ramli, Department of Medical Microbiology and Immunology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455923 Fax: +603-91456671 Email: ramliza@ppukm.ukm.edu.my', NULL, 1088, 64, 72, 'Alcalá, L., Sánchez-Cambronero, L., Catalán, M.P., Sánchez-Somolinos, M., Peláez, M.T., Marín, M., Bouza, E. 2008. Comparison of three commercial methods for rapid detection of Clostridium difficiletoxins A and B from fecal specimens. J Clin Microbiol 46(11): 3833–35.\r\nAslam, S., Musher, D.M. 2006. An update on diagnosis, treatment andprevention of Clostridium difficile-associated disease. Gastroenterol Clin North Am 35(2): 315–35.\r\nBartlett, J.G. 1996. Management of Clostridium difficile infection and other antibiotic-associated diarrhoeas. Eur J Gastroenterol Hepatol 8(11): 1054-61.\r\nBartlett, J.G. 2008. Historical perspectives on studies of Clostridium difficileand C. difficileinfection. Clin Infect Dis 46(Suppl 1): 4–11.\r\nBliss, D.Z., Johnson, S., Savik, K., Clabots, C.R., Willard, K., Gerding D.N. 1998.Acquisition of Clostridium difficile and Clostridium difficile–associated diarrhea in hospitalized patients receiving tube feeding. Ann Intern Med 129(12):1012–19.\r\nChung, C.H., Wu, C.J., Lee, H.C., Yan, J.J., Chang, C.M., Lee, N.Y., Chen, P.L., Lee, C.C., Hung, Y.P., Ko, W.C. 2010. Clostridium difficile infection at a medical center in Southern Taiwan: incidence, clinical features and prognosis. J Microbiol Immunol Infect 43(2): 119–25.\r\nCohen, S.H., Gerding, D.N., Johnson, S., Kelly, C.P., Loo, V.G., McDonald, L.C., Pepin, J., Wilcox, M.H. 2010.Clinical practice guidelines for Clostridium difficile infection in adults: 2010 update by the Society for Healthcare Epidemiology of America (SHEA) and the Infectious Diseases Society of America (IDSA). 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Analysis of prevalence, risk factors and molecular epidemiology of Clostridium difficile infectionin Kuwait over a 3-year period. Anaerobe 16(6): 560-65.\r\nKim, H.H., Kim, Y.S., Han, D.S., Kim, Y.H., Kim, W.H., Kim, J.S., Kim, H., Kim, H.S., Park, Y.S., Song, H.J., Shin, S.J., Yang, S.K., Ye, B.D., Eun, C.S., Lee, K.M., Lee, S.H., Jang, B.I., Jung, S.A., Cheon, J.H, Choi, C.H., Huh, K. 2014. Clinical differences in Clostridium difficile infection based on age: a multicenter study. Scand J Infect Dis 46(1): 46–51.\r\nKyne, L., Sougioultzis, S., McFarland, L.V., Kelly, C.P. 2002. Underlying disease severity as a major riskfactor for nosocomial Clostridium difficile diarrhea. Infect Control Hosp Epidemiol 23(11): 653-9.\r\nLai, C.C., Lin, S.H., Tan, C.K., Liao, C.H., Huang, Y.T., Hsueh, P.R. 2013. Clinical manifestations of Clostridium difficile infection in a medical center in Taiwan. J Microbiol Immunol Infect, pii: S1684-1182(13)00111-4.\r\nLee, Y.C., Wang, J.T., Chen, A.C., Sheng, W.H., Chang, S.C., Chen, Y.C. 2012. Changing incidence and clinical manifestations of Clostridium difficile-associated diarrhea detected by combination of glutamate dehydrogenase and toxin assay in Northern Taiwan. J Microbiol Immunol Infect 45(4): 287-95.\r\nLim, P.L., Barkham, T.M., Ling, L.M., Dimatatac, F., Alfred, T., Ang, B. 2008. Increasing incidence of Clostridium difficile–associated disease, Singapore. Emerg Infect Dis 14(9): 1487-89.\r\nManian, F.A., Aradhyula, S., Greisnauer, S., Senkel, D., Setzer, J., Wiechens, M., Meyer, P.L. 2007. Is it Clostridium difficile infection or something else? A case-control study of 352 hospitalized patients with new-onset diarrhea. South Med J 100(8): 782-6.\r\nMcFarland, L.V. 1998. Epidemiology, risk factors and treatments for antibiotic-associated diarrhea. 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(1116, 1108, 'Clinical Decision Making Ability of Nursing Students', 'Assoc Prof. Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456250 Fax: +603-91456683 Email: angieho88@gmail.com / ho@ppukm.ukm.edu.my', NULL, 1088, 73, 80, 'Aitken, L.M., Marshall, A.P., Elliott, R., McKinley, S. 2009. Critical care nurses’ decision making: sedation assessment and management in intensive care. J Clin Nurs 18(1): 36-45.\r\nBjörkström, M.E., Athlin, E.E., Johansson, I.S. 2008. Nurses’ development of professional self--from being a nursing student in a baccalaureate programme to an experienced nurse. J Clin Nurs 17(10): 1380-91.\r\nBucknall, T. 2003. The clinical landscape of critical care: Nurses’ decision making. J Adv Nurs 43(3): 310-19.\r\nCaldwell, L.M., Tenofsky, L.M., Nugent, E. 2010. Academic and clinical immersion in an accelerated nursing program to foster learning in the adult student. Nurs Educ Perspect 31(5): 294-7.\r\ndel Bueno, D. 2005. A crisis in critical thinking. Nurs Educ Perspect 26(5): 278-82.\r\nGordon, C.J., Frotjold, A., Fethney, J., Green, J., Hardy, J., Maw, M., Buckley, T. 2013. The effectiveness of simulation-based blood pressure training in preregistration nursing students. Simul Healthc 8(5): 335-40.\r\nHorton, C.D., DePaoli, S., Hertach, M., Bower, M. 2012. Enhancing the effectiveness of nurse preceptors. J Nurses Staff Dev 28(4): E1-7.\r\nJenkins, H. M. 1985. Improving clinical decision making in nursing. J Nurs Educ 24(6): 242-3.\r\nKoehler, C. J. 2004. Nursing process mapping replaces nursing care plans. In Fuszard’s innovative teaching strategies in nursing (3rd ed.). Edited by A. J. Lowenstein, & M. J. Bradshaw. Sudbury: Jones and Bartlett; 303-13.\r\nSmith, A., Lollar, J., Mendenhall, J., Brown, H., Johnson, P., Roberts, S. 2013. Use of multiple pedagogies to promote confidence in triage decision making: A pilot study. J Emerg Nurs 39(6): 660-6.\r\nStanding, M. 2007. Clinical decision-making skills on the developmental journey from student to Registered Nurse: A longitudinal injury. J Adv Nurs 60(3): 257-69.\r\nWiles, L.L., Simko, L.C., Schoessler, M. 2013. What do I do now?: clinical decision making by new graduates. J Nurses Prof Dev 29(4): 167-72.'),
(1120, 1112, 'Intraosseous Cannulation', 'Bala Krishnian s/o Muniandy, Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455495 Fax: +603-91456577 Email: blrmsh@yahoo.com', NULL, 1088, 81, 84, 'American Heart Association in collaboration with the International Liaison Committee on Resuscitation 2005a. Guidelines for cardiopulmonary resuscitation and emergency cardiovascular care: Management of cardiac arrest. Circulation 112: 58-66.\r\nAmerican Heart Association in collaboration with the International Liaison Committee on Resuscitation 2005b. Guidelines for cardiopulmonary resuscitation and emergency cardiovascular care. Pediatric advanced life support. Circulation 112: 167-87.\r\nBiarent, D., Bingham, R., Richmond, S., Maconochie, I., Wyllie, J., Simpson, S., Nunez, A.R., Zideman, D. 2005. European Resuscitation Council Guidelines for resuscitation 2005: Paediatric life support. Resuscitation 67(Suppl 1): S97-S133.\r\nCostantino, T.G., Fojtik, J.P. 2003. Success rate of peripheral IV catheter insertion by emergency physicians using ultrasound guidance. Acad Emerg Med 10: 487-91.\r\nCostantino, T.G., Parikh, A.K., Satz, W.A., Fojtik, J.P. 2005. Ultrasonography-guided peripheral intravenous access versus traditional approaches in patients with difficult intravenous access. Ann Emerg Med 46(5): 456-61.\r\nLapostolle, F., Catineau, J., Garrigue, B., Monmarteau, V., Houssaye, T., Vecci, I., Tréoux, V., Hospital, B., Crocheton, N., Adnet, F. 2007. Prospective evaluation of peripheral venous access difficulty in emergency care. Intensive Care Med 33(8): 1452-7.\r\nLewis, F.R. 1986. Prehospital intravenous fluid therapy: physiologic computer modelling. J Trauma 26(9): 804-11.\r\nMinville, V., Pianezza, A., Asehnoune, K., Cabardis, S., Smail, N. 2006. Prehospital intravenous line placement assessment in the French emergency system: a prospective study. Eur J Anaesthesiol 23(7): 594-7.\r\nPfister, C.A., Egger, L., Wirthmuller, B., Greif, R. 2008. Structured training in intraosseous infusion to improve potentially life-saving skills in pediatric emergencies: results of an open prospective national quality development project over 3 years. Pediatr Anesth 18: 223-9.\r\nTurner, J., Nicholl, J., Webber, L., Cox, H., Dixon, S., Yates, D. 2000. A randomised controlled trial of prehospital intravenous fluid replacement therapy in serious trauma. Health Technol Assess 4(31): 1-57.'),
(1122, 1114, 'TB Prostate', 'Dr. Tan Geok Chin, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455362 Fax: +603-91456676 Email: tan_geok_chin@yahoo.com /tangc@ppukm.ukm.edu.my', NULL, 1088, 85, 88, 'Chawla, A., Chawla, K., Reddy, S., Arora, N., Bairy, I., Rao, S., Hegde, P., Thomas, J. 2012. Can Tissue PCR Augment the Diagnostic Accuracy in Genitourinary Tract Tuberculosis? Urol Int 88(1): 34–8.\r\nEastwood, J.B., Corbishley, C.M., Grange, J.M. 2001.Tuberculosis and the kidney. J Am Soc Nephrol 12(6): 1307-14.\r\nGhaleb, K., Afifi, M., El-Gohary, M. 2013.Assessment of diagnostic techniques of urinary tuberculosis Mediterr J Hematol Infect Dis 5(1): e2013034.\r\nGlobal Tuberculosis Report. 2013. World Health Organization Press. Switzerland. http://www.who.int/tb/country/en/index.html. [8 February 2014].\r\nGow, J.G. 1998. Genitourinary tuberculosis. In: Campbell’s Urology. 7th edition. Edited by Walsh PC, Retik AB, Vaughan EA, Wein AJ. Philadelphia: WB Saunders; 817-8.\r\nGupta, N., Mandal, A.K., Singh, S.K. 2008. Tuberculosis of the prostate and urethra: A review. Indian J Urol 24(3): 388-91.\r\nMortier, E., Pouchot, J., Girard, L., Boussougan, Y., Vinceneux, P. 1996. Assessment of urine analysis for the diagnosis of tuberculosis. BMJ 312(7022): 27–8.\r\nRai, A., Pahwa, H., Jain, V., Misra, S. 2009.Management of Genito-Urinary Tuberculosis. The Internet Journal of Surgery 23(1): DOI: 10.5580/173a.\r\nSporer, A., Auerbach, O. 1978. Tuberculosis of prostate. Urology 11(4): 362–5.'),
(1124, 1116, 'Orbital Cellulitis', 'Prof. Dr. Ismail Mohd Saiboon, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455491 Fax: +603-91456577 Email: fadzmail69@yahoo.com.my', NULL, 1088, 89, 93, 'Chaudhry, I.A., Shamsi, F.A., Elzaridi, E., Al- Rasheed, W., Al- Amri, A., Arat, Y.O. 2008. Inpatient preseptal cellulitis: experience from a tertiary eye care centre. Br J Ophthalmol 92(10): 1337-41.\r\nChaudhry, I.A., Al-Rashed, W., Arat, Y.O. 2012. The hot orbit: orbital cellulitis. Middle East Afr J Ophthalmol 19(1): 34-42.\r\nDuke-Elder, S., MacFaul, P.A. 1974. The ocular adnexa: part 2. Lacrimal orbital and para orbital diseases. In: System of Ophthalmology. Edited by Duke-Elder S. London: Henry Kimpton; 859–89.\r\nManiglia, A.J., Goodwin, W.J., Arnold, J.E., Ganz, E. 1989. Intracranial abscesses secondary to nasal, sinus, and orbital infections in adults and children. Arch Otolaryngol Head Neck Surg 115(12): 1424–9.\r\nMair, M.H., Geley, T., Judmaier, W., Gassner, I. 2002. Using orbital sonography to diagnose and monitor treatment of acute swelling of the eyelids in pediatric patients. AJR Am J Roentgenol 179(6): 1529-34.\r\nReddy, S.C, Tajunisah, I., Low, K.P., Karmila, A.B. 2008. Prevalence of Eye Diseases and Visual Impairment In Urban Population - A Study from University of Malaya Medical Centre. Malaysian Family Physicians 3(1): 25-8.\r\nSchramm, V.L., Myers, E.N., Kennerdell, J.S. 1978. Orbital complications of acute sinusitis: evaluation, management and outcome. Otolaryngology 86(2): 221-30.\r\nSuhaili, D.N., Goh, B.S., Gendeh, B.S. 2010. A ten year retrospective review of orbital complications secondary to acute sinusitis in children. Med J Malaysia 65(1): 49-52.\r\nWilliams, S.R., Carruth, J.A. 1992. Orbital infection secondary to sinusitis in children: diagnosis and management. Clin Otolaryngol Allied Sci 17(6): 550-7.'),
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(1133, 1125, 'Variant Common Hepatic and Middle Colic Artery', 'Assoc. Prof. Prajna Paramita Samanta, Department of Anatomy, School of Medical Sciences & Research, Sharda University, Plot No. 32-34, Knowledge Park 3, Greater Noida, 201306 U.P, India. Tel: +91-9818063477 Fax: +91-0120-4060200 Email: psprajna@gmail.com', NULL, 1088, 100, 103, 'Adachi, B. 1928. A Coeliaca Arteriensystem der Japaner. Bd. II Verlag Der Kaiserlich-Japanischen. Universitant zu Kyoto; 18-71.\r\nBenton, R.S., Cotter, W.B. 1963. A hitherto undocumented variation of the inferior mesenteric artery in man. Anat Rec 145: 171-3.\r\nCavdar, S., Gurbuz, J., Zeybek, A., Sehirli, U., Abhik, L., Ozdogmus, O. 1998. A variation of celiac trunk. Kaibogaku Zasshi 73(5): 505-8.\r\nChen, C.Y., Lee, R.C., Tseng, H.S., Chaing, J.H., Hwang, J.I., Teng, M.M. 1998. Normal and variant anatomy of the hepatic arteries: angiographic experience. Zhonghua Yi Xue Za Zhi 61(1): 17-23.\r\nChitra, R. 2010. Clinically relevant variations of the coeliac trunk. Singapore Med J 51(3): 216-9.\r\nChou, C.K., Mak, C.W., Hou, C.C., Chang, J.M., Tzeng, W.S. 1997. CT of the mesenteric vascular anatomy. Abdom Imaging 22(5): 477-82.\r\nHiatt, J.R., Gabbay, J., Busuttil, R.W. 1994. Surgical anatomy of the hepatic arteries in 1000 cases. Ann Surg 220(1): 50-2.\r\nStandring, S. 2005. Gray’s anatomy: The anatomical basis of clinical practice. Philadelphia: Elsevier Churchill Livingstone; 768-72.\r\nUgurel, M. S., Battal, B., Bozlar, U., Nural, M.S., Tasar, M., Ors, F., Saglam, M., Karademir, I. 2010. Anatomical variations of hepatic arterial system, coeliac trunk and renal arteries: an analysis with multidetector CT angiography. Br J Radiol 83(992): 661-7.\r\nYildirim, M., Celik, H.H., Yildiz, Z., Tatar, I., Aldur, M.M. 2004. The middle colic artery originating from the celiac trunk. Folia Morphol (Warsz) 63(3): 363-5.'),
(1166, 1158, 'Quantitative Ultrasound (QUS) and Bone Assessment', 'Siti Zulfarina Mohamed, Department of Pharmacology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Center, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: 03-91459547 Fax: 03-91459547 Email: szulfarinamohamed@gmail.com', 1077, 1155, 1, 10, 'Malaysian Osteoporosis Society. 2001. Malaysian clinical practices guidelines on management of osteoporosis. Malaysian Osteoporosis Society. 1-53\r\nBachrach, L. K., Sills, I. N. 2011. Clinical report-bone densitometry in children and adolescents. Pediatrics 127(1): 189-94.\r\nBauer, D.C., Gluer, C.C., Cauley, J.A., Vogt, T.M., Ensrud, K.E., Genant, H.K., Black, D.M. 1997. Broadband Ultrasound Attenuation Predicts Fractures Strongly and Independently of Densitometry in Older Women: A Prospective Study. Arch Intern Med 157(6): 629-634\r\nChin, K.Y., Ima Nirwana, S. 2013. 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INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
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(1231, 1223, 'A Morphometric Analysis of Intercondylar Notch of Femur with Emphasis on Its Clinical Implications', 'Ameet Jha. Gandaki Medical College, Tribhuvan University, Lekhnath, Kaski - 33700, Nepal. Tel: +977-61-561718, Fax: +977-61-561768, Email: ameetjha1980@yahoo.com', NULL, 1218, 103, 108, 'Alizadeh, A., Kiavash, V. 2008. Mean intercondylar notch width index in cases with and without anterior cruciate ligament tears. Iran J Radiol 5(4): 205-8.\r\nAnderson, A.F., Lipscomb, A.B., Liudahl, K.J., Addlestone, R.B. 1987. Analysis of the intrcondylar notch by computed tomography. Am J Sports Med 15(6): 547-52.\r\nDavis, T.J., Shelbourne, K.D., Klootwyk, T.E. 1999. Correlation of the intercondylar notch width of the femur to the width of the anterior and posterior cruciate ligaments. Knee Surg Sports Traumatol Arthrosc 7(4): 209-14.\r\nFarrow, L.D., Chen, M.R., Cooperman, D.R., Victoroff, B.N., Goodfellow, D.B. 2007. Morphology of the femoral intercondylar notch. J Bone Joint Surg Am 89(10): 2150-5.\r\nGood, L., Odensten, M., Gillquist, J. 1991. Intercondylar notch measurements with special reference to anterior cruciate ligament surgery. Clin Orthop Relat Res (263): 185-9.\r\nLeon, H.O., Blanco, C.E., Guthrie, T.B., Martinez, O.J. 2005. Intercondylar notch stenosis in degenerative arthritis of the knee. Arthroscopy 21(3): 294-302.\r\nHerzog, R.J., Silliman, J.F., Hutton, K., Rodkey, W.G., Steadman, J.R. 1994. Measurements of the intercondylar notch by plain film radiography and magnetic resonance imaging. Am J Sports Med 22(2): 204-10.\r\nLombardo, S., Sethi, P.M., Starkey, C. 2005. Intercondylar notch stenosis is not a risk factor for anterior cruciate ligament tears in professional male basketball players: an 11-year prospective study. Am J Sports Med 33(1): 29-34.\r\nMiller, R.H. 2008. Knee Injuries. In: Campbell’s Operative Orthopaedics. Volume 3. 11th edition. Mosby: Elsevier; 2496–2550.\r\nPalmer, I. 2007. On the injuries to the ligaments of the knee joint: a clinical study. 1938. Clin Orthop Relat Res 454: 17-22.\r\nRavichandran, D., Melani, R. 2010. Morphology of the intercondylar notch and its clinical significance. International Journal of Anatomical Sciences 1: 26-30.\r\nSchickendantz, M.S., Weiker, G.G. 1993. The predictive value of radiographs in the evaluation of unilateral and bilateral anterior cruciate ligament injuries. Am J Sports Med 21(1): 110-3.\r\nShepstone, L., Rogers, J., Kirwan, J.R., Silverman, B.W. 2001. Shape of the intercondylar notch of the human femur: a comparison of osteoarthritic and non-osteoarthritic bones from a skeletal sample. Ann Rheum Dis 60(10): 968–73.\r\nSouryal, T.O., Moore, H.A., Evans, J.P. 1988. Bilaterality in anterior cruciate ligament injuries: associated intercondylar notch stenosis. Am J Sports Med 16(5): 449-54.\r\nSouryal, T.O., Freeman, T.R. 1993. Intercondylar notch size and anterior cruciate ligament injuries in athletes. A prospective study. Am J Sports Med 21(4): 535-9.\r\nvan Eck, C.F., Martins, C.A., Vyas, S.M., Celentano, U., van Dijk, C.N., Fu, F.H. 2010. Femoral intercondylar notch shape and dimensions in ACL-injured patients. Knee Surg Sports Traumatol Arthrosc 18(9): 1257-62\r\nWada, M., Tatsuo, H., Baba, H., Asamoto, K., Nojyo, Y. 1999. Femoral intercondylar notch measurements in osteoarthritic knees. Rheumatology (Oxford) 38(6): 554-8.\r\nWilliams, P.L., Bannister, L.H., Berry, M.M. 1995. Femur. In: Gray’s Anatomy. 38th edition. Edinburgh and London: ELBS with Churchill Livingstone; 680.'),
(1234, 1226, 'Cardiovascular Fitness among COPD Patients', 'Vikram Mohan, Department of Physiotherapy, Faculty of Health Sciences, Universiti Teknologi MARA (UiTM), Bandar Puncak Alam, 42300 Puncak Alam, Selangor, Malaysia. Tel: +603-32584367 Fax: +603-32584599 Email: vikkipt@gmail.com', NULL, 1218, 109, 113, 'Ansari, M., Javadi, H., Pourbehi, M., Mogharrabi, M., Rayzan, M., Semnani, S., Jallalat, S., Amini, A., Abbaszadeh, M., Barekat, M., Nabipour, I., Assadi, M. 2012. The association of rate pressure product (RPP) and myocardial perfusion imaging (MPI) findings: a preliminary study. Perfusion 27(3): 207-13.\r\nBorghi-Silva, A., Arena, R., Castello, V., Simões, R.P., Martins, L.E., Catai, A.M., Costa, D. 2009. Aerobic exercise training improves autonomic nervous control in patients with COPD. Respir Med 103(10): 1503-10.\r\nGigliothi, F., Coli, C., Bianchi, R., Romagnoli, I., Lannin, B., Binazzi, B., Scano, G. 2003. Exercise training improves external dyspnoea in patients with COPD: evidence of the role of medical factors. Chest 123(6): 1794-1802.\r\nKamahara, K., Homma, T., Naito, A., Matsumura, T., Nakayama, M., Kadono, K., Kai, S., Inoue, M., Satoh, H., Sekizawa, K. 2004. Circuit training for elderly patients with chronic obstructive pulmonary disease: a preliminary study. Arch Gerontol Geriatr 39(2): 103–10.\r\nKongsgaard, M., Backer, V., Jørgensen, K., Kjaer, M., Beyer, N. 2004. Heavy resistance training increases muscle size, strength and physical function in elderly male COPD-patients- a pilot study. Respir Med 98(10): 1000–7.\r\nLeão da Silva, P., de Mello, M.T., Cheik, N.C., Sanches, P.L., Munhoz da Silveira Campos, R., Carnier, J., Inoue, D., do Nascimento, C.M., Oyama, L.M., Tock, L., Tufik, S., Dâmaso, A.R. 2012. Reduction in the leptin concentration as a predictor of improvement in lung function in obese adolescents. Obes Facts 5(8): 806-20.\r\nLellamo, F., Manzi, V., Caminiti, G., Vitale, C., Massaro, M., Cerrito, A., Rosano, G., Volterrani, M. 2014. Validation of rate of perceived exertion-based exercise training in patients with heart failure: Insights from autonomic nervous system adaptations. Int J Cardiol 176(2): 394-8.\r\nLopez, A.D., Shibuya, K., Rao, C. 2006. Chronic obstructive pulmonary disease: current burden and future projections. Eur Respir J 27(2): 397–412.\r\nMador, M. J., Bozkanat, E., Aggarwal, A., Shaffer, M., Thomas, J. K. 2004. Endurance and Strength Training in Patients With COPD. Chest 125(6): 2036-45.\r\nRam, F.S., Robinson, S.M., Black, P.N. 2006. Treatment review-Physical training improves cardiovascular fitness and life style in Asthma and does not decrease lung function. Evidence based Nurs 9(2): 52-65.\r\nRochester, C.L. 2003. Exercise training in chronic obstructive pulmonary disease. J Rehabil Res Dev 40(5 Suppl 2): 59-80.'),
(1241, 1233, 'Emotional Disorder', 'Dr. Aida Jaffar, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456117 Fax: +603-91456580 Email: aida.jaffar@gmail.com', NULL, 1218, 114, 123, 'Abdul Ghani, H.M. 2008. Stress and depression among medical students: A cross sectional study at a medical college in Saudi Arabia. Pak J Med Sci 24: 12–17.\r\nAida, J., Azimah, M.N., Mohd Radzniwan, A.R., T Iryani, M.D., Ramli, M., Khairani, O. 2010. Barriers to the utilization of primary care services for mental health problems among adolescents in a secondary school in Malaysia. Malaysian Family Physician 5(1): 31-35.\r\nBenjamin J.S. & Virginia A.S. 2005. Kaplan & Sadock’s Pocket Handbook of Clinical Psychiatry 4th ed. Philadelphia: Lippincott Williams & Wilkins; 148.\r\nBunevicius, A., Katkute, A., Bunevicius, R. 2008. Symptoms of anxiety and depression in medical students and in humanities students: relationship with big-five personality dimensions and vulnerability to stress. Int J Soc Psychiatry 54(6): 494-501.\r\nDrapeau, A., Marchand, A., Beaulieu-Prévost, D. 2012. Epidemiology of psychological distress. In Mental Illnesses - Understanding, Prediction and Control. http://www.intechopen.com/books/mental-illnesses-understanding-prediction-and-control/epidemiology-of-psychological-distress. [4th November 2014].\r\nDesrosiers, A., Miller, L. 2007. Relational spirituality and depression in adolescent girls. Inc J Clin Psychol 63(10): 1021–37.\r\nEisenberg, D., Golberstein, E., Gollust, S.E. 2007. Help-seeking and access to mental health care in a university student population. Med Care 45(7): 594-601.\r\nFan, A.P., Kosik, R.O., Mandell, G.A., Tran, D.T., Cheng, H.M., Chen, C.H., Su, T.P., Chiu, A.W. 2012. Suicidal ideation in medical students: who is at risk? Ann Acad Med Singapore 41(9): 337-82.\r\nGould, M.S., Munfakh, J.L., Lubell, K., Kleinman, M., Parker, S. 2002. Seeking help from the internet during adolescence. J Am Acad Child Adolesc Psychiatry 41(10): 1182-9.\r\nLPPKN. 2008. Focus on Parenting and Reproductive Health. National Population and Family Development Board Malaysia. http://www.thestar.com.my/story.aspx/?file=%2f2008%2f1%2f10%2fcentral%2f19932625&sec=central [29 March 2014].\r\nRamli, M., Muhd Ariff, F., Zaini, Z. 2007. Translation, validation and psychometric properties of Bahasa Malaysia version of the depression anxiety and stress scales (DASS). ASEAN Journal Of Psychiatry 8(2): 82-9.\r\nRosenthal, J.M., Okie, S. 2005. White coat, mood indigo-depression in medical school. N Engl J Med 353(11): 1085-8.\r\nShah, M., Hasan, S., Malik, S., Sreeramareddy, C.T. 2010. Perceived Stress, sources and severity of stress among medical undergraduates in a Pakistani medical school. BMC Med Educ 10: 2.\r\nShamsuddin, K., Fadzil, F., Ismail, W.S., Shah, S.A., Omar, K., Muhammad, N.A., Jaffar, A., Ismail, A., Mahadevan, R. 2013. Correlates of depression, anxiety and stress among Malaysian university students. Asian J Psychiatr 6(4): 318-23.\r\nSherina, M.S., Lekhraj, R., Nadarajan, K. 2003. Prevalence of emotional disorder among medical students in a Malaysian university. Asia Pac Fam Med 2: 213-7.\r\nTjia, J., Givens, J.L., Shea, J.A. 2005. Factors associated with undertreatment of medical student depression. J Am Coll Health 53(5): 219-24.\r\nTuisku, V., Pelkonen, M., Karlsson, L., Kiviruusu, O., Holi, M., Ruuttu, T., Punamäki, R.L., Marttunen, M. 2006. Suicidal ideation, deliberate self-harm behaviour and suicide attempts among adolescent outpatients with depressive mood disorders and comorbid axis I disorders. Eur Child Adolesc Psychiatry 15(4): 199–206.\r\nYusoff, M.S., Abdul Rahim, A.F., Baba, A.A., Ismail, S.B., Mat Pa, M.N., Esa, A.R. 2013. Prevalence and associated factors of stress, anxiety and depression among prospective medical students. Asian J Psychiatr 6(2): 128-133.\r\nZaid, Z.A., Chan, S.C., Ho, J.J. 2007. Emotional disorders among medical students in a Malaysian private medical school. Singapore Med J 48(10): 895-9.'),
(1248, 1240, 'Static Lactate and Lactate Clearance with 28 Day Sepsis Mortality', 'Tan Toh Leong, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91458224 Fax: +603-91456577 Email: sebastianttl@yahoo.co.uk.', NULL, 1218, 124, 133, 'Aduen, J., Bernstein, W.K., Khastgir, T., Miller, J., Kerzner, R., Bhatiani, A., Lustgarten, J., Bassin, A.S., Davison, L., Chernow, B. 1994. The use and clinical importance of a substrate-specific electrode for rapid determination of blood lactate concentrations. JAMA 272(21): 1678–85.\r\nBakker, J., Gris, P., Coffernils, M., Kahn, R.J., Vincent, J.L. 1996. Serial blood lactate levels can predict the development of multiple organ failure following septic shock. Am J Surg 171(2): 221–6.\r\nBakker, J. 2001. Lactate: may I have your votes please? 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Serum lactate as a predictor of mortality in emergency department patients with infection. Ann Emerg Med 45(5): 524–8.\r\nValenza, F., Aletti, G., Fossali, T., Chevallard, G., Sacconi, F., Irace, M., Gattinoni, L. 2005. Lactate as a marker of energy failure in critically ill patients: hypothesis. Crit Care 9(6): 588-93.\r\nVarpula, M., Tallgren, M., Saukkonen, K., Voipio-Pulkki, L.M., Pettila, V. 2005. Hemodynamic variables related to outcome in septic shock. Intensive Care Med 31(8): 1066–71.\r\nWang, X.Z., Lü, C.J., Gao, F.Q., Li, X.H., Yan, W.F., Ning, F.Y. 2006. Efficacy of goal-directed therapy in the treatment of septic shock. Zhongguo Wei Zhong Bing Ji Jiu Yi Xue 18(11): 661-4.\r\nWalker, C.A., Griffith, D.M., Gray, A.J., Datta, D., Hay, A.W. 2013. Early lactate clearance in septic patients with elevated lactate levels admitted from the emergency department to intensive care: time to aim higher? J Crit Care 28(5): 832-7.'),
(1252, 1244, 'Long-term Outcome of Asymmetry ROP', 'Dr. Mushawiahti Mustapha, Department of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +60391455981 Fax: +60391737856 Email: drmusha@yahoo.com/drmusha@gmail.com', NULL, 1218, 134, 138, 'Al-Ghamdi, A., Albiani, D.A., Hodge, W.G., Clarke, W.N. 2004. Myopia and astigmatism in retinopathy of prematurity after treatment with cryotherapy or laser photocoagulation. Can J Ophthalmol 39(5): 521-5.\r\nAzad, R., Chandra, P., Patwardhan, S.D., Gupta, A. 2010. Profile of asymmetrical retinopathy of prematurity in twins. Indian J Ophthalmol 58(3): 209-11.\r\nCryotherapy for Retinopathy of Prematurity Cooperative Group. 1988. Multicenter trial of cryotherapy for retinopathy of prematurity: preliminary results. Arch Ophthalmol 106(4): 471-9.\r\nGarcia-Valenzuela, E., Kaufman, L.M. 2005. High myopia associated with retinopathy of prematurity is primarily lenticular. J AAPOS 9(2): 121-8.\r\nHardy, R.J., Palmer, E.A., Schaffer, D.B., Phelps, D.L., Davis, B.R., Cooper, C.J. 1997. Outcome-based management of retinopathy of prematurity. Multicenter Trial of Cryotherapy for Retinopathy of Prematurity Cooperative Group. J AAPOS 1(1): 46-54.\r\nKatoch, D., Sanghi, G., Dogra, M.R., Beke, N., Gupta, A. 2011. Structural sequelae and refractive outcome 1 year after laser treatment for type 1 prethreshold retinopathy of prematurity in Asian Indian eyes. Indian J Ophthalmol 59(6): 423-6.\r\nNg, E.Y., Connolly, B.P., McNamara, J.A., Regillo, C.D., Vander, J.F., Tasman, W. 2002. A comparison of laser photocoagulation with cryotherapy for threshold retinopathy of prematurity at 10 years: part 1. Visual function and structural outcome. Ophthalmology 109(5): 928-34.\r\nPalmer, E.A., Flynn, J.T., Hardy, R.J., Phelps, D.L., Phillips, C.L., Schaffer, D.B., Tung, B. 1991. Incidence and early course of retinopathy of prematurity. The Cryotherapy for Retinopathy of Prematurity Cooperative Group. Ophthalmology 98(11): 1628-40.\r\nRahi, J.S., Cable, N. 2003. Severe visual impairment and blindness in children in the UK. Lancet 362(9393): 1359-65.\r\nTodd, D., Kennedy, J., Roberts, V., John, E. 1990. Risk factors in progression beyond stage 2 retinopathy of prematurity. Aust N Z J Ophthalmol 18(1): 57-60.\r\nVanderVeen, D.K., Bremer, D.L., Fellows, R.R., Hardy, R.J., Neely, D.E., Palmer, E.A., Rogers, D.L., Tung, B., Good, W.V. 2011. Prevalence and course of strabismus through age 6 years in participants of the Early Treatment for Retinopathy of Prematurity randomized trial. J AAPOS 15(6): 536-40.'),
(1255, 1247, 'Managing ‘Schizophrenia in the Midst of Schizophrenia’', 'Prof. Dr. Marhani Midin, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91457428 Fax: +6-03-91456681 Email:marhanimidin@yahoo.com.', NULL, NULL, 139, 142, 'Coldwell, C.M., Bender, W.S. 2007. The effectiveness of assertive community treatment for homeless populations with severe mental illness: a meta-analysis. Am J Psychiatry 164(3): 393–9.\r\nDahlan R, Midin M, Sidi H. 2013. Remission of symptoms among schizophrenia patients receiving assertive community treatment (act ) in Malaysia: One year follow-up. Sains Malaysiana 42(3): 389-97.\r\nDixon, L.B., Dickerson, F., Bellack, A.S., Bennett, M., Dickinson, D., Goldberg, R.W., Lehman, A., Tenhula, W.N., Calmes, C., Pasillas, R.M., Peer, J., Kreyenbuhl, J. 2010. The 2009 Schizophrenia PORT Psychosocial Treatment Recommendations and Summary Statements. Schizophr Bull 36(1): 48–70.\r\nLambert, M., Bock, T., Schöttle, D., Golks, D., Meister, K., Rietschel, L., Bussopulos, A., Frieling, M., Schödlbauer, M., Burlon, M., Huber, C.G., Ohm, G., Pakrasi, M., Chirazi-Stark, M.S., Naber, D., Schimmelmann, B.G. 2010. Assertive community treatment as part of integrated care versus standard care: a 12-month trial in patients with first- and multiple-episode schizophrenia spectrum disorders treated with quetiapine immediate release (ACCESS trial). J Clin Psychiatry 71(10): 1313-23.\r\nRuzanna, Z., Marhani, M. 2008. Assertive Community Treatment (ACT) For Patients With Severe Mental Illness: Experience In Malaysia. Malaysian Journal of Psychiatry 17(1): 1-7.'),
(1258, 1250, 'Managing ‘Schizophrenia in the Midst of Schizophrenia’', 'Prof. Dr. Marhani Midin, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91457428 Fax: +6-03-91456681 Email:marhanimidin@yahoo.com.', NULL, 1218, 139, 142, 'Coldwell, C.M., Bender, W.S. 2007. The effectiveness of assertive community treatment for homeless populations with severe mental illness: a meta-analysis. Am J Psychiatry 164(3): 393–9.\r\nDahlan R, Midin M, Sidi H. 2013. Remission of symptoms among schizophrenia patients receiving assertive community treatment (act ) in Malaysia: One year follow-up. Sains Malaysiana 42(3): 389-97.\r\nDixon, L.B., Dickerson, F., Bellack, A.S., Bennett, M., Dickinson, D., Goldberg, R.W., Lehman, A., Tenhula, W.N., Calmes, C., Pasillas, R.M., Peer, J., Kreyenbuhl, J. 2010. The 2009 Schizophrenia PORT Psychosocial Treatment Recommendations and Summary Statements. Schizophr Bull 36(1): 48–70.\r\nLambert, M., Bock, T., Schöttle, D., Golks, D., Meister, K., Rietschel, L., Bussopulos, A., Frieling, M., Schödlbauer, M., Burlon, M., Huber, C.G., Ohm, G., Pakrasi, M., Chirazi-Stark, M.S., Naber, D., Schimmelmann, B.G. 2010. Assertive community treatment as part of integrated care versus standard care: a 12-month trial in patients with first- and multiple-episode schizophrenia spectrum disorders treated with quetiapine immediate release (ACCESS trial). J Clin Psychiatry 71(10): 1313-23.\r\nRuzanna, Z., Marhani, M. 2008. Assertive Community Treatment (ACT) For Patients With Severe Mental Illness: Experience In Malaysia. Malaysian Journal of Psychiatry 17(1): 1-7.');
INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
(1264, 1256, 'Multiple Acute Aortic Dissection', 'Dr.Nik Ritza Kosai Nik Mahmood, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456208 Fax: +603-91456202 Email: nikkosai@yahoo.co.uk', NULL, 1218, 143, 149, 'Chen, S., Yei, F., Zhou, L., Luo, J., Zhang, J., Shan, S., Tian, N., Kwan, T.W. 2006. Endovascular stent-grafts treatment in acute aortic dissection (type B): clinical outcomes during early, late, or chronic phases. Catheter Cardiovasc Interv 68(2): 319-25.\r\nEggebrecht, H., Nienaber, C.A., Neuhäuser, M., Baumgart, D., Kische, S., Schmermund, A., Herold, U., Rehders, T.C., Jakob, H.G., Erbel, R. 2006. Endovascular stent-graft placement in aortic dissection: a meta-analysis. Eur Heart J 27(4): 489-98.\r\nEstrera, A.L., Miller, C.C., Safi, H.J., Goodrick, J.S., Keyhani, A., Porat, E.E., Achouh, P.E., Meada, R., Azizzadeh, A., Dhareshwar, J., Allaham, A. 2006. Outcomes of medical management of acute type B aortic dissection. Circulation 114(1 Supply): I384–9.\r\nGolledge, J., Eagle, K.A. 2008. Acute aortic dissection. Lancet 372(9632): 55-66.\r\nHagan, P.G., Nienaber, C.A., Isselbacher, E.M., Bruckman, D., Karavite, D.J., Russman, P.L., Evangelista, A., Fattori, R., Suzuki, T., Oh, J.K., Moore, A.G., Malouf, J.F., Pape, L.A., Gaca, C., Sechtem, U., Lenferink, S., Deutsch, H.J., Diedrichs, H., Marcos y Robles, J., Llovet, A., Gilon, D., Das, S.K., Armstrong, W.F., Deeb, G.M., Eagle, K.A. 2000. The International Registry of Acute Aortic Dissection (IRAD): new insights into an old disease. JAMA 283(7): 897–903.\r\nHiratzka, L.F., Bakris, G.L., Beckman, J.A., Bersin, R.M., Carr, V.F., Casey, D.E., Eagle, K.A., Hermann, L.K., Isselbacher, E.M., Kazerooni, E.A., Kouchoukos, N.T., Lytle, B.W., Milewicz, D.M., Reich, D.L., Sen, S., Shinn, J.A., Svensson, L.G., Williams, D.M. 2010. 2010 ACCF/AHA/AATS/ACR/ASA/SCA/SCAI/SIR/STS/SVM guidelines for the diagnosis and management of patients with Thoracic Aortic Disease: a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines, American Association for Thoracic Surgery, American College of Radiology, American Stroke Association, Society of Cardiovascular Anesthesiologists, Society for Cardiovascular Angiography and Interventions, Society of Interventional Radiology, Society of Thoracic Surgeons, and Society for Vascular Medicine. Circulation 121(13): e266-369.\r\nHirst, A.E., Johns, V.J., Kime, S.W. 1958. Dissecting aneurysm of the aorta: a review of 505 cases. Medicine (Baltimore) 37(3): 217–79.\r\nJanuzzi, J.L., Isselbacher, E.M., Fattori, R., Cooper, J.V., Smith, D.E., Fang, J., Eagle, K.A., Mehta, R.H., Nienaber, C.A., Pape, L.A. 2004. Characterizing the young patient with aortic dissection: results from the International Registry of Aortic Dissection (IRAD). J Am Coll Cardiol 43(4): 665–9.\r\nKhan, I.A., Nair, C.K. 2002. Clinical, diagnostic and management perspectives of aortic dissection. Chest 122(1): 311-28.\r\nKlompas, M. 2002. Does this patient have an acute thoracic aortic dissection? JAMA 287(17): 2262–72.\r\nLedbetter, S., Stuk, J.L., Kaufman, J.A. 1999. Helical (spiral) CT in the evalution of emergent thoracic aortic syndromes. Traumatic aortic rupture, aortic aneurysm, aortic dissection, intramural hematoma, and penetrating atherosclerotic ulcer. Radiol Clin North Am 37(3): 575-89.\r\nLeurs, L.J., Bell, R., Degrieck, Y., Thomas, S., Hobo, R., Lundbom, J. 2004. Endovascular treatment of thoracic aortic diseases: combined experience from the EUROSTAR and United Kingdom Thoracic Endograft registries. J Vasc Surg 40(4): 670-9.\r\nRoberts, C.S., Roberts, W.C. 1991. Aortic dissection with the entrance tear in the descending thoracic aorta. Analysis of 40 necropsy patients. Ann Surg 213(4): 356–68.\r\nSheikh, A.S., Ali, K., Mazhar, S. 2013. Acute aortic syndrome. Circulation 128(10): 1122–7.\r\nShiga, T., Wajima, Z., Apfel, C.C., Inoue, T., Ohe, Y. 2006. Diagnostic accuracy of transesophageal echocardiography, helical computed tomography, and magnetic resonance imaging for suspected thoracic aortic dissection: systematic review and meta-analysis. Arch Intern Med 166(13): 1350–6.\r\nSommer, T., Fehske, W., Holzknecht, N., Smekal, A.V., Keller, E., Lutterbey, G., Kreft, B., Kuhl, C., Gieseke, J., Abu-Ramadan, D., Schild, H. 1996. Aortic dissection: a comparative study of diagnosis with spiral CT, multiplanar transesophageal echocardiography and MR imaging. Radiology 199(2): 347-52.\r\nSvensson, L.G., Crawford, E.S. 1992. Aortic dissection and aortic aneurysm surgery: clinical observations, experimental investigations, and statistical analyses. Part II. Curr Probl Surg 29(12): 913–1057.\r\nSvensson, L.G., Labib, S.B., Eisenhauer, A.C., Butterly, J.R. 1999. Intimal tear without hematoma: an important variant of aortic dissection that can elude current imaging techniques. Circulation 99(10): 1331– 6.\r\nSvensson, L.G., Nadolny, E.M., Kimmel, W.A. 2002. Multimodal protocol influence on stroke and neurocognitive deficit prevention after ascending/arch aortic operations. Ann Thorac Surg 74(6): 2040–6.\r\nVon Kodolitsch, Y., Schwartz, A.G., Nienaber, C.A. 2000. Clinical prediction of acute aortic dissection. 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(1267, 1259, 'Orogastric Lavage for Fatal Poisoning', 'Dr. Nik Azlan Nik Muhamad, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455703 Fax: 03-91456577 Email: nikazlanmuhamad@hotmail.com', NULL, 1218, 150, 154, 'American Board of Applied Toxicology. http://www.clintox.org/ABAT_Main.cfm [15 October 2014].\r\nBenson B.E., Hoppu K., Troutman W.G., Bedry R., Erdman A., Höjer J., Mégarbane B., Thanacoody R., Caravati E.M. 2013. Position paper update: gastric lavage for gastrointestinal decontamination. Clin Toxicol (Phila) 51(3):140–6.\r\nCanadian Association of Poison Control Centre. http://www.capcc.ca/ [14 October 2014]\r\nEddleston M., Haggalla S., Reginald K., Sudarshan K., Senthilkumaran M., Karalliedde L., Ariaratnam A., Sheriff M.H., Warrell D.A., Buckley N.A. 2007. The hazards of gastric lavage for intentional self-poisoning in a resource poor location. Clin Toxicol (Phila) 45(2):136-43.\r\nLarkin GL, Claassen C. 2007. Trends in emergency department use of gastric lavage for poisoning in the united stated 1993-2003, Clin Toxicol (Phila) 45(2):164-8.\r\nMerigian K.S., Woodard M., Hedges J.R., Roberts J.R., Stuebing R., Rashkin M.C. 1990. Prospective evaluation of gastric emptying in self poisoning patients. Am J Emerg Med 8(6):479-83.\r\nPond S.M., Lewis-Driver D.J., Williams G.M., Green A.C., Stevenson N.W. 1995. Gastric emptying in acute overdose: A prospective randomized controlled trial . Med J Aust 163(7):345-9.\r\nThompson A.M., Robins J.B., Prescott L.F. 1987. Changes in cardiorespiratory function during gastric lavage for drug overdose. Hum Toxicol 6(3):215-8.\r\nVale J.A., Kulig K., American Academy of Clinical Toxicology, European Association of Poisons Centres and Clinical Toxicologist. 2004. Position Paper: Gastric Lavage. J Toxicol Clin Toxicol 42(7): 933-43.'),
(1275, 1267, 'Vein of Galen Malformation', 'Dr. Reynu Rajan, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456202 Fax: +603-91456684 Email: dr.reynu@gmail.com', NULL, 1218, 155, 156, NULL),
(1316, 1307, 'Acne Disability among Medical Students', 'Dr. Leelavathi Muthupalaniappen, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456123 Fax: +603-91456680 Email: drleelaraj@gmail.com', NULL, 1301, 1, 9, 'Ali, G., Mehtab, K., Sheikh, Z.A., Ali, H.G., Abdel Kader, S., Mansoor, H., Altaf, S., Qamar, S., Khwaja, S.S. 2010. Beliefs and perceptions of acne among a sample of students from Sindh Medical College, Karachi. J Pak Med Assoc 60(1): 51-4.\r\nCheng, C.E., Irwin, B., Mauriello, D., Liang, L., Pappert, A., Kimball, A.B. 2010. Self-reported acne severity, treatment, and belief patterns across multiple racial and ethnic groups in adolescent students. Pediatr Dermatol 27(5): 446-52.\r\nGoncalves, G., Amado, J.M., Matos, M., Massa, A. 2012. The prevalence of acne among a group of Portuguese medical students. J Eur Acad Dermatol Venereol 26(4): 514-7.\r\nKatsambas, A.D., Lotti, T.M. 2003. European Handbook of Dermatological Treatments. 2nd edition. Berlin: Springer-Verlag; 3-9.\r\nKilkenny, M., Merlin, K., Plunkett, A., Marks, R. 1998. The prevalence of common skin conditions in Australian school students: 3. Acne vulgaris. Br J Dermatol 139(5): 840–5.\r\nKokandi, A. 2010. Evaluation of acne quality of life and clinical severity in acne female adults. Dermatol Res Pract pii: 410809.\r\nKoo, J. 1995. The psychosocial impact of acne: patients’ perceptions. J Am Acad Dermatol 32(5 Pt 3): S26-30.\r\nLasek, R.J., Chren, M.M. 1998. Acne vulgaris and the quality of life of adult dermatology patients. Arch Dermatol 134(4): 454–8.\r\nLaw, M.P., Chuh, A.A., Lee, A., Molinari, N. 2009. Acne Prevalence and Beyond: acne disability and its predictive factors among Chinese late adolescents in Hong Kong. Clin Exp Dermatol 35(1):16-21.\r\nMotley, R.J., Finlay, A.Y. 1992. Practical use of a disability index in the routine management of acne.Clin Exp Dermatol 17(1): 1-3.\r\nNewton, J.N., Mallon, E., Klassen, A., Ryan, T.J., Finlay, A.Y. 1997. The effectiveness of acne treatment: an assessment by patients of the outcome of therapy. Br J Dermatol 137(4): 563–7.\r\nPicardi, A., Abeni, D., Melchi, CF., Puddu, P., Pasquini, P. 2000. Psychiatric morbidity in dermatological outpatients: an issue to be recognized. Br J Dermatol 143(5): 983–91.\r\nSmithard, A., Glazebrook, C., William, H.C. 2001. Acne prevalence, knowledge about acne and psychological morbidity in mid-adolescence: a community-based study. Br J Dermatol 145(2): 274-9.\r\nSuh, D.H., Shin, J.W., Mi, S.U., Lee, D.H., Yoon, M.Y., Kim, N.I., Kye, Y.C., Lee, E.S., Ro, Y.S., Kim, K.J. 2008. Treatment-Seeking behaviors and related epidemiological features in Korean acne patients. J Korean Med Sci 23(6): 969-74.\r\nTan, H.H., Tan, A.W., Barkham, T., Yan, X.Y., Zhu, M. 2007. Community-based study of acne vulgaris in adolescents in Singapore. Br J Dermatol 157(3): 547-51.\r\nTan, J.K., Tang, J., Fung, K., Gupta, A.K., Thomas, D.R., Sapra, S., Lynde, C., Poulin Y., Gulliver, W., Sebaldt, R.J. 2007. Development and validation of a comprehensive acne severity scale. J Cutan Med Surg 11(6): 211–16.\r\nYap, F.B. 2012. Cardiff acne disability index in Sarawak, Malaysia. Ann Dermatol 24(2): 158–61'),
(1322, 1313, 'Immunohistochemistry in Breast Cancer Biopsy', 'Dr. Reena Rahayu Md Zin, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +60391455359 Fax: +60391456676 Email: reenarahayu@ppukm.ukm.edu.my', NULL, 1301, 10, 16, 'Arnedos, M., Nerurkar, A., Osin, P., A’Hern, R., Smith, I.E., Dowsett, M. 2009. Discordance between core needle biopsy (CNB) and excisional biopsy (EB) for estrogen receptor (ER), progesterone receptor (PgR) and HER2 status in early breast cancer (EBC). Ann Oncol 20(12): 1948-52.\r\nArnould, L., Roger, P., Macgrogan, G., Chenard, M.P., Balaton, A., Beauclair, S., Penault-Llorca, F. 2012. Accuracy of HER2 status determination on breast core-needle biopsies (immunohistochemistry, FISH, CISH and SISH vs FISH). Mod Pathol 25(5): 675-82.\r\nBurge,C.N., Chang, H.R., Apple, S.K. 2006. Do the histologic features and results of breast cancer biomarker studies differ between core biopsy and surgical excision specimens? Breast 15(2): 167-72.\r\nConnor, C.S., Tawfik, O.W., Joyce, A.J., Davis, M.K., Mayo, M.S., Jewell, W.R. 2002. A comparison of prognostic tumor markers obtained on image-guided breast biopsies and final surgical specimens. Am J Surg 184(4): 322-4.\r\nD’Alfonso, T., Liu, Y.F., Monni, S., Rosen, P.P., Shin, S.J. 2010. Accurately assessing her-2/neu status in needle core biopsies of breast cancer patients in the era of neoadjuvant therapy: emerging questions and considerations addressed. Am J Surg Pathol 34(4): 575-81.\r\nHonkoop, A.H., Pinedo, H.M., De Jong, J.S., Verheul, H.M., Linn, S.C., Hoekman, K., Wagstaff, J., van Diest, P.J.1997. Effects of chemotherapy on pathologic and biologic characteristics of locally advanced breast cancer. Am J Clin Pathol 107(2): 211-8.\r\nLi, S., Yang, X., Zhang, Y., Fan, L., Zhang, F., Chen, L., Zhou, Y., Chen, X., Jiang, J. 2012. Assessment accuracy of core needle biopsy for hormone receptors in breast cancer: a meta-analysis. Breast Cancer Res Treat 135(2): 325-34.\r\nRicci, M.D., Calvano Filho, C.M., Oliveira Filho, H.R., Filassi, J.R., Pinotti, J.A., Baracat, E.C. 2012. Analysis of the concordance rates between core needle biopsy and surgical excision in patients with breast cancer. Rev Assoc Med Bras 58(5): 532-6.\r\nSutela, A., Vanninen, R., Sudah, M., Berg, M., Kiviniemi, V., Rummukainen, J., Kataja, V., Kärjä, V. 2008. Surgical specimen can be replaced by core samples in assessment of ER, PR and HER-2 for invasive breast cancer. Acta Oncol 47(1): 38-46.\r\nTamaki, K., Sasano, H., Ishida, T., Miyashita, M., Takeda, M., Amari, M., Tamaki, N., Ohuchi, N. 2010. Comparison of core needle biopsy (CNB) and surgical specimens for accurate preoperative evaluation of ER, PgR and HER2 status of breast cancer patients. Cancer Sci 101(9): 2074-9.\r\nUy, G.B., Laudico, A.V., Carnate, J.M., Lim, F.G., Fernandez, A.M., Rivera, R.R., Mapua, C.A., Love, R.R. 2010. Breast cancer hormone receptor assay results of core needle biopsy and modified radical mastectomy specimens from the same patients. Clin Breast Cancer 10(2): 154-9.\r\nWai, C.J., Al-Mubarak, G., Homer, M.J., Goldkamp, A., Samenfeld-Specht, M., Lee, Y., Logvinenko, T., Rothschild, J.G., Graham, R.A. 2013. A modified triple test for palpable breast masses: the value of ultrasound and core needle biopsy. Ann Surg Oncol 20(3): 850-5.\r\nWood, B., Junckerstorff, R., Sterrett, G., Frost, F., Harvey, J., Robbins, P. 2007. A comparison of immunohistochemical staining for oestrogen receptor, progesterone receptor and HER-2 in breast core biopsies and subsequent excisions. Pathology 39(4): 391-5.\r\n'),
(1326, 1317, 'Wobble Board Training in Unstable Ankle Joint', 'Sundaraganesh Kandasamy, Division of Physical Medicine and Rehabilitation, Rajah Muthiah Medical College and Hospital, Annamalai University, Chidambaram 608002, India. Tel: +919894403020 Fax: +914144 238499 Email: sundarganesh.k@gmail.com\r\n', NULL, 1301, 17, 22, 'Azeem, Z,, Dhawan, V., Zutsh, K. 2010. Comparison of functional balance training and wobble board training on subjects with functional ankle instability. Br J Sports Med 44: i15-i16.\r\nBernier, J.N., Perrin, D.H. 1998. Effect of coordination training on proprioception of the functionally unstable ankle. J Orthop Sports Phys Ther 27(4): 264-75.\r\nBohannon, R.W., Larkin, P.A., Cook, A.C., Gear, J., Singer, J. 1984. Decrease in timed balance test scores with aging. Phys Ther 64(7): 1067-70.\r\nClark, V.M., Burdern, A.M. 2005. A 4 week wobble board exercise programme improved muscle onset latency and perceived stability in individuals with a functionally unstable ankle.  Phys Ther Spor 6(4):181-7.      \r\nDoherty, C., Delahunt, E., Caulfield, B., Hertel, J., Ryan, J., Bleakley, C. 2014. The incidence and prevalence of ankle sprain injury: a systematic review and meta-analysis of prospective epidemiological studies. Sports Med 44(1):123-40. \r\nDuncan, P.W., Weiner. D.K., Chandler, J., Studenski, S. 1990. Functional reach: a new clinical measure of balance. J Gerontol 45(6): M192-7. \r\nFreeman, M.A., Dean, M.R., Hanham, I.W. 1965. The etiology and prevention of functional instability of the foot. J Bone Joint Surg Br 47(4): 678-85.\r\nHoffman, M., Payne, V.G. 1995. The effects of proprioceptive ankle disk training on healthy subjects. J Orthop Sports Phys Ther 21(2): 90-3.\r\nHübscher, M., Zech, A., Pfeifer, K., Hänsel, F., Vogt, L., Banzer, W. 2010. Neuromuscular training for sports injury prevention: a systematic review. Med Sci Sports Exerc 42(3): 413-21. \r\nLee, A., Lin, W.H., Huang, C. 2006. Impaired proprioception and poor static postural control in subjects with functional instability of the ankle. J Exerc Sci Fit 4: 117-25. \r\nMattacola, C.G., Lloyd, J.W. 1997. Effects of a 6-week strength and proprioception training program on measures of dynamic balance: a single-case design. J Athl Train 32(2): 127-35.\r\nMcGuine, T.A., Keene, J.S. 2006. The effect of a balance training program on the risk of ankle sprains in high school athletes. Am J Sports Med 34(7): 1103-11.\r\nRoss, S.E,. Guskiewicz, K.M. 2004. Examination of static and dynamic postural stability in individuals with functionally stable and unstable ankles. Clin J Sport Med 14(6): 332-8.\r\nWaterman, B.R., Owens, B.D., Davey, S., Zacchilli, M.A., Belmont, P.J.. 2010. The epidemiology of ankle sprains in the United States. J Bone Joint Surg Am 92(13): 2279-84.'),
(1329, 1320, 'Radiation Retinopathy in Nasopharyngeal Carcinoma', 'Norshamsiah Md Din, Department of Ophthalmology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +6013-3444010 Fax: +603-91737836 Email: shamsiahdr@hotmail.com', NULL, 1301, 23, 31, 'Branch Vein Occlusion Study Group. 1986. Argon laser scatter photocoagulation for prevention of neovascularization and vitreous hemorrhage in branch vein occlusion. A randomized clinical trial. Arch Ophthalmol 104(1): 34-41.\r\nBrown, G.C., Shields, J.A., Sanborn, G., Augsburger, J.J., Savino, P.J., Schatz, N.J. 1982. Radiation retinopathy. Ophthalmology 89(12): 1494–1501.\r\nChan, A.T., Teo, P.M., Johnson, P.J. 2002. Nasopharyngeal carcinoma. Ann Oncol 13(7): 1007-15.\r\nHempel, M., Hinkelbein, W. 1993. Eye sequelae following external irradiation. Recent Results Cancer Res 130: 231–6.\r\nHo, J.H. 1978. An epidemiologic and clinical study of nasopharyngeal carcinoma. Int J Radiat Oncol Biol Phys 4(3-4): 183-205.\r\nGupta, P., Meisenberg, B., Amin, P., Pomeranz, H.D. 2001. Radiation retinopathy: the role of pentoxifylline. Retina 21(5): 545-7.\r\nKinyoun, J.L., Lawrence, B.S., Barlow, W.E. 1996. Proliferative radiation retinopathy. Arch Ophthalmol 114(9): 1097-1100.\r\nLee, A,W., Sze, W.M., Au, J.S., Leung, S.F., Leung, T.W., Chua, D.T., Zee, B.C., Law, S.C., Teo, P.M., Tung, S.Y., Kwong, D.L., Lau, W.H. 2005. Treatment results for nasopharyngeal carcinoma in the modern era: the Hong Kong experience. Int J Radiat Oncol Biol Phys 61(4): 1107-16.\r\nNoble, K.G., Kupersmith, M.J. 1984. Retinal vascular remodeling in radiation retinopathy. Br J Ophthalmol 68(7): 475-8.\r\nParsons, J.T. 2004. Radiation toxicity to the visual system. J Neuroophthalmol 24(3): 193-4.\r\nParsons, J.T., Fitzgerald, C.R., Hood, C.I., Ellingwood, K.E., Bova, F.J., Million, R.R. 1983. The effects of irradiation on the eye and the optic nerve. Int J Radiat Oncol Biol Phy 9(5): 609-22.\r\nPuri, D.R., Chou, W., Lee, N. 2005. Intensity-modulated radiation therapy in head and neck cancers: dosimetric advantages and update of clinical results. Am J Clin Oncol  28(4): 415-23.         \r\nRosenblatt, E., Brook, O.R., Erlich, N., Miller, B., Joachims, H.Z., Kuten, A. 2003. Late visual and auditory toxicity of radiotherapy for nasopharyngeal carcinoma. Tumori 89(1): 68-74.\r\nTakeda, A., Shigematsu, N., Suzuki, S., Fujii, M., Kawata, T., Kawaguchi, O., Uno, T., Takano, H., Kubo, A., Ito, H. 1999. Late retinal complications of radiation therapy for nasal and paranasal malignancies: relationship between irradiated-dose area and severity. Int J Radiat Oncol Biol Phys 44(3): 599–605. '),
(1332, 1323, 'Post Stroke Functional Status', 'Bala Krishnian R.Muniandy, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455495 Fax: +60391456577 Email: blrmsh@yahoo.com', NULL, 1301, 32, 36, 'Bendz, M. 2003. The first years of rehabilitation after a stroke-from two perspectives. Scand J Caring Sci 17(3): 215-222.\r\nBurton, C.R. 2000. Re-thinking stroke rehabilitation: the Corbin and Strauss chronic Illness trajectory framework. J Adv Nurs 32(3): 595-602. \r\nElmstahl, S., Sommer, M., Hagberg, B. 1996. A 3 – year follow-up of stroke patient: relationships between activities of daily living and personality characteristics. Arch Gerontol Geriatr 22(3): 233-244.\r\nEriksson, M., Norrving, B., Terent, A., Stegmayr, B. 2008. Functional outcome 3 months after stroke predicts long-term survival. Cerebrovasc Dis 25(5): 423-429.\r\nHeart and Stroke Foundation of Ontario & Registered Nurses Association of Ontario. 2005. Stroke assessment across the continuum of care. Toronto: Heart and Stroke foundation of Ontario and Registered Nurses Association of Ontario.http://www.rnao.org/bestpractices/PDF [8 May 2008] \r\nInternet Stroke Center. 2008. What is a stroke? http://www.strokecenter.org/patients/about.htm [29 May 2008].\r\nKapral, M.K., Silver, F.L., Richards, J.A., Lindsay, M.P., Fang, J., Shi, S., Hill, M.D., Phillips, S.J., Robertson, A., Tu, J.V. 2005.  Registry of the Canadian Stroke Network. Report on 2004/05. Toronto: Institute for Clinical Evaluative Sciences. http://www.ontariostrokenetwork.ca/wp-content/uploads/2014/06/Full-Report1.pdf [10 May 2008]\r\nRosamond, W., Flegal, K., Furie, K., Go, A., Greenlund, K., Haase, N., Hailpern, S.M., Ho, M., Howard, V., Kissela, B., Kittner, S., Lloyd-Jones, D., McDermott, M., Meigs, J., Moy, C., Nichol, G., O’Donnell, C., Roger, V., Sorlie, P., Steinberger, J., Thom, T., Wilson, M., Hong, Y. 2008. Heart disease and stroke statistics--2008 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. Circulation 117(4): e25-146.\r\nWolfe, C.D., Taub, N.A., Woodrow, E.J., Burney, P.G. 2002. Assessment of scales of disability and handicap for stroke patients. Stroke 22(10): 1242-1244.\r\nWorld Health Organization. 2002. Towards a Common Language for Functioning, Disability, and Health. The International Classification of Functioning, Disability and Health (ICF). Geneva: World Health Organization. http://www.jefferson.edu/content/dam/tju/JIEC/files/WHO%20-%20ICF.pdf [15 May 2008]\r\n'),
(1342, 1333, 'Nutrient Foramina of Metatarsals', 'Murlimanju BV, Department of Anatomy, Kasturba Medical College, Manipal University, Mangalore – 575004, India. Tel: 91824 2211746 Fax: 91824 2421283 Email: flutemist@gmail.com\r\n', NULL, 1301, 37, 47, 'Brookes, M. 1963. Cortical vascularization and growth in foetal tubular bones. J Anat 97(Pt 4): 597-609.\r\nCollipal, E., Vargas, R., Parra, X., Silva, H., Del Sol, M. 2007. Diaphyseal nutrient foramina in the femur, tibia and fibula bones. Int J Morphol 25(2): 305-8. \r\nGreen, D.P. 1988. Operative hand surgery. 2nd edition. New York: Churchill Livingstone; 1248.\r\nHashimoto, I., Abe, Y., Morimoto, A., Kashiwagi, K., Goishi, K., Nakanishi, H. 2014. Limb salvage and vascular augmentation by microsurgical free flap transfer for treatment of neuropathic diabetic foot ulcers. J Med Invest 61(3-4): 325-32.\r\nHenderson, R.G. 1978. The position of the nutrient foramen in the growing tibia and femur of the rat. J Anat 125(Pt 3): 593-9.\r\nHenry, S.L., Wei, F.C. 2010. Thumb reconstruction with toe transfer. J Hand Microsurg 2(2): 72-78. \r\nHughes, H. 1952. The factors determining the direction of the canal for the nutrient artery in the long bones of mammals and birds. Acta Anat (Basel) 15(3): 261-80.\r\nKizilkanat, E., Boyan, N., Ozsahin, E.T., Soames, R., Oguz, O. 2007. Location, number and clinical significance of nutrient foramina in human long bones. Ann Anat 189(1): 87-95.\r\nLacroix, P. 1951. Organization of Bones. London: J. and A. Churchill; 138.\r\nLutken, P. 1950. Investigation into the position of the nutrient foramina and the direction of the vessel canals in the shafts of the humerus and femur in man. Acta Anatomica 9: 57-68.\r\nMalukar, O., Joshi, H. 2011. Diaphysial nutrient foramina in long bones and miniature long bones. NJIRM 2(2): 23-6. \r\nMcKeon, K.E., Johnson, J.E., McCormick, J.J., Klein, S.E. 2013. The intraosseous and extraosseous vascular supply of the fifth metatarsal: implications for fifth metatarsal osteotomy. Foot Ankle Int 34(1): 117–23.\r\nMurlimanju, B.V., Prashanth, K.U., Prabhu, L.V., Saralaya, V.V., Pai, M.M., Rai, R. 2011. Morphological and topographical anatomy of nutrient foramina in human upper limb long bones and their surgical importance. Rom J Morphol Embryol 52(3): 859-62.\r\nMysorekar, V.R. 1967. Diaphysial nutrient foramina in human long bones. J Anat 101(Pt 4): 813-22.\r\nPatake, S.M., Mysorekar, V.R. 1977. Diaphysial nutrient foramina in human metacarpals and metatarsals. J Anat 124(Pt 2): 299-304.\r\nPayton, C.G. 1934. The position of the nutrient foramen and direction of the nutrient canal in the long bones of the madder-fed pig. J Anat 68(Pt 4): 500-10.\r\nPho, R.W.H. 1988. Microsurgical technique in orthopaedics. 1st edition. Scotland: Butterworth & Co. Ltd; 128.\r\nPrashanth, K.U., Murlimanju, B.V., Prabhu, L.V., Kumar, C.G., Pai, M.M., Dhananjaya, K.V.N. 2011. Morphological and topographical anatomy of nutrient foramina in the lower limb long bones and its clinical importance. Australas Med J 4(10): 530-7.\r\nPremchandran, D., Murlimanju, B.V., Prabhu, L.V., Saralaya, V.V., Kumari, A., Rao, K.A., Prashanth, K.U., Rai, A. 2013. Topography and morphological anatomy of nutrient foramina in human metacarpal bones and their clinical implications. Clin Ter 164(4): 295-300.\r\nSchwalbe, G. 1876. Zeitschrift fur Anatomie und Entwicklungsgeschichte. 1: 307-52.\r\nShrimankar, D.P.S., Trivedi, D.J., Kariya, V.B. 2010. A study of nutrient foramina of the metacarpal bones. NJIRM 1(2): 16-21.\r\nSingh, I. 1960. Variations in the metatarsal bones. J Anat 94: 345-50. \r\nSnell, R.S. 2007. Clinical Anatomy by Systems. Baltimore: Lippincott Williams and Wilkins; 358.\r\nSpanio, S., Wei, F.C., Coskunfirat, O.K., Lin, C.H., Lin, Y.T. 2005. Symmetry of vascular pedicle anatomy in the first web space of the foot related to toe harvest: clinical observations in 85 simultaneous bilateral second-toe transfer patients. Plast Reconstr Surg 115(5): 1325-7.\r\nZitelli, B.J., McIntire, S.C., Nowalk, A.J. 2012. Zitelli and Davis’ Atlas of Pediatric Physical Diagnosis.  6th edition. Philadelphia, PA: Elsevier Saunders; 908.\r\n'),
(1344, 1335, 'Depression among Adolescent', 'Norfazilah Ahmad, Department of Community Health, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91458781 Fax: +603-91456670 Email: norfazilah@ppukm.ukm.edu.my', NULL, 1301, 48, 57, 'Adelusi, J.O., Coker, A.O. 2013. The relationship between family structure and psychopathology among adolescents and young adults in Lagos, South-Western Nigeria. Nigerian Journal of Psychiatry 11(1): 10-4. \r\nAnda, D., Baroni, S., Boskin, L., Buchwald, L., Morgan, J., Ow, J., Gold, J.S., Weiss, R. 2000. Stress, stressors and coping among high school students. Child Youth Serv Rev 22(6): 441- 63. \r\nBachanas, P., Kaslow, N. 2001. Depressive disorders in Hughes, J., Greca, A., Conoley, J. Handbook of Psychological Services for Children and Adolescents. New York: Oxford University Press; 231-4.\r\nBeck, A.,T. 1967. 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(1349, 1340, 'Patient Education Package for Pain', 'Ho Siew Eng, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456250 Fax: +603-91738254 Email: ho@ppukm.ukm.edu.my', NULL, 1301, 58, 65, 'Al Samaraee, A., Rhind, G., Saleh, U., Bhattacharya, V. 2010. Factors contributing to poor post-operative abdominal pain management in adult patients: a review. Surgeon 8(3): 151–8. \r\nBoyd-Seale, D., Wilkie, D.J., Kim, Y.O., Suarez, M.L., Lee, H., Molokie, R., Zhao, Z., Zong, S. 2010. Pain barriers: psychometrics of a 13-Item questionnaire. Nurs Res 59(2): 93–101. \r\nBozimowski, G. 2012. Patient perceptions of pain management therapy: a comparison of real-time assessment of patient education and satisfaction and registered nurse perceptions. Pain Manag Nurs 13(4): 186–93.\r\nBrown, C., Constance, K., Bédard, D., Purden, M. 2013. Colorectal surgery patients’ pain status, activities, satisfaction, and beliefs about pain and pain management. Pain Manag Nurs 14(4): 184-92.\r\nCostello, M., Thompson, S. 2014. Preventing opioid misuse and potential abuse: the nurse’s role in patient education. Pain Manag Nurs pii: S1524-9042(14)00155-6, in press.\r\nCaumo, W., Schmidt, A.P., Schneider, C.N., Bergmann, J., Iwamoto, C.W., Bandeira, D., Ferreira, M.B. 2001. Risk factors for preoperative anxiety in adults. Acta Anaesthesiol Scand 45(3): 298–307. \r\nDihle, A., Bjølseth, G., Helseth, S. 2006. The gap between saying and doing in postoperative pain management. J Clin Nurs 15(4): 469–79.\r\nHo, S.E., Choy, Y.C., Rozainiee, A. 2009. Survey of nurses’ knowledge and attitude regarding post operative pain management at a teaching hospital in Malaysia. Medicine & Health 4(1): 47-52.\r\nHo, S.E., Ho, C.C., Pang Yuen, H.,  Lexshimi, R., Choy, Y.C., Jaffar, M.Z., Cardosa, M., Das, S. 2013. A study of knowledge and attitudes of registered nurses towards pain managment in an urban hsopital. Clin Ter 164(3): 215-9.\r\nKapoor, S., Thorn, B.E. 2014. Healthcare use and prescription of opioids in rural residents with pain. Rural Remote Health 14(3): 2879. \r\nLim, R. 2008. Improving cancer pain management in Malaysia. Oncology 74(Suppl 1): 24-34.\r\nLin, L.Y., Wang, R.H. 2005. Abdominal surgery, pain and anxiety: preoperative nursing intervention. J Adv Nurs 51(3): 252–60. \r\nMcCaffrey, M., Ferrell, B.R. 1997. Nurses’ knowledge of pain assessment and management: How much progress have we made? J Pain Symptom Manage 14(3): 175–88.\r\nOrtiz, J., Wang, S., Elayda, M.A., Tolpin, D.A. 2015. Preoperative patient education: can we improve satisfaction and reduce anxiety?  Rev Bras Anestesiol 65(1): 7-13.\r\nPatel, R., Biros, M.H., Moore, J., Miner, J.R. 2014. Gender differences in patient described pain, stress, and anxiety among patients undergoing treatment for painful conditions in the emergency department. Acad Emerg Med 21(12): 1478-84.\r\nPolomano, R.C., Dunwoody, C.J., Krenzischek, D.A., Rathmell, J.P. 2008. Perspective on pain management in the 21st century. Pain Manage Nurs 9(1 Suppl): S3-10.\r\nTaylor, A., Stanbury, L. 2009. A review of postoperative pain management and the challenges. Curr Anaesth Crit Care 20: 188–94.\r\n'),
(1352, 1343, 'Music Therapy and Post-Operative Pain', 'Santhna Letchmi Panduragan, Department of Nursing, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia.  Tel: +603-91456264 Fax: +603-91456683 Email: santhna@ppukm.ukm.edu.my', NULL, 1301, 66, 79, 'Allred, K.D., Byers, J.F., Sole, M.L. 2010. The effect of music on postoperative pain and anxiety. Pain Manag Nurs 11(1): 15-25.\r\nAmerican Music Therapy Association Inc -Silver Spring, Maryland. http://www.musictherapy.org/ [6 October 2014]\r\nBunt L. Music therapy: An art beyond words. 2004: Routledge. http://samples.sainsburysebooks.co.uk/9781317815334_sample_581732.pdf [6 October 2014]\r\nDahlen, L., Zimmerman, L., Barron, C. 2006. Pain perception and its relation to functional status post total knee arthroplasty: a pilot study. Orthop Nurs 25(4): 264-270.\r\nEbneshahidi, A., Mohseni, M. 2008. 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(1355, 1346, 'Focal Cardiac Concussion', 'Tan Toh Leong, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel:+603-91458224  Fax: +603-91456577 Email: sebastianttl@yahoo.co.uk \r\n', NULL, 1301, 80, 85, 'Maron, B.J., Poliac, L.C., Kaplan, J.A., Mueller, F.O. 1995. Blunt Impact to the Chest Leading to Sudden Death from Cardiac Arrest during Sports Activities. N Engl J Med 333:337-42.\r\nBishop, L.H., Estes, E.H., McIntosh, H.D. 1956. The electrocardiogram as a safeguard in pericardiocentesis. J Am Med Assoc 162(4):264-5.\r\nDettmeyer, R.B. 2011. Histopathology of Selected Trauma. Forensic histopathology: Fundamentals and perspectives. Springer-Verlag Berlin Heidelberg: Science & Business Media; 37-65.\r\nKaplan, J.A., Karofsky, P.S., Volturo, G.A. 1993. Commotiocordis in two amateur ice hockey players despite the use of commercial chest protectors: case reports. J Trauma 34(1):151-3.\r\nKoehler, S.A., Shakir, A., Ladham, S., Rozin, L., Omalu, B., Dominick, J., Wecht, C.H. 2004. Cardiac Concussion: Definition, differential diagnosis, and cases presentation and the legal ramification of a misdiagnosis. Am J Forensic Med Pathol 25(3):205-8.'),
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(6045, 6036, 'Statin: Hope for a New Agent to Treat Osteoporosis', 'Department of Pharmacology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia', NULL, 6034, 88, 89, 'Abdul-Majeed, S., Mohamed, N., Soelaiman, I.N. 2012. Effects of Tocotrienol and Lovastatin Combination on Osteoblast and Osteoclast Activity in Estrogen-Deficient Osteoporosis. Evid Based Complement Alternat Med. 2012:960742.\r\nIbrahim, N’., Khamis. M.F., Mod Yunoh, M.F., Abdullah, S., Mohamed, N., Shuid, A.N. 2014. Targeted Delivery of Lovastatin and Tocotrienol to Fracture Site Promotes Fracture Healing in Osteoporosis Model: Micro-Computed Tomography and Biomechanical Evaluation. PLoS One 9(12):e115595.\r\nMundy, G., Garrett, R., Harris, S., Chan, J., Chen, D., Rossini, G., Boyce, B., Zhao, M., Gutierrez, G. 1999. Stimulation of bone formation in vitro and in rodents by statins. Science 286(5446):1946-9.\r\nSalari, P. and M. Abdollahi, 2011. A Systematic Review on the Relation between use of Statins and Osteoporosis. International Journal of Pharmacology 7(2): 180-8.');
INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
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Effects of food processing on the stability of food allergens. Biotechnol Adv 23(6): 423-9.\r\nShahnaz, M., Gendeh, B.S., Nasuruddin, A. 2001. Skin test reactivity to inhalant food allergens in patients with allergic rhintis. Int Med Res J 5(2): 69–73.\r\nShen, Y., Cao, M.J., Cai, Q.F., Su, W.J., Yu, H.L., Ruan, W.W., Liu, G.M. 2011. Purification, cloning, expression and immunological analysis of Scylla serrata arginine kinase, the crab allergen. J Sci Food Agric 91(7): 1326-35.\r\nShriver, S.K., Yang, W. 2011. Thermal and nonthermal methods for food allergen control. Food Eng Rev 3: 26-43.\r\nSuma, Y., Ishizaki, S., Nagashima, Y., Lu, Y., Ushio, H., Shiomi, K. 2007. Comparative analysis of barnacle tropomyosin: divergence from decapod tropomyosins and role as a potential allergen. Comp Biochem Physiol B Biochem Mol Biol 147(2): 230-6.\r\nYadzir, Z.H., Misnan, R., Abdullah, N., Bakhtiar, F., Arip, M., Murad, S. 2010. 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(6053, 6044, 'Post-Stroke Depression', 'Bala Krishnian M, Department of Emergency Medicine, Faculty of Medicine,Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455495 Fax: +603-91456577 Email: blrmsh@yahoo.com', NULL, 6034, 98, 102, 'Beck, A.T., Ward, C.H., Mendelson, M., Mock, J. Erbaugh, J. 1961. An Inventory for Measuring Depression. Arch Gen Psychiatry 4(6): 561-71.	\r\nBendz, M. 2003. The first years of rehabilitation after a stroke from two perspectives. Scand J Caring Sci 17(3): 215-22.\r\nBourgeois, J.A., Hilty, D.M., Chang, C.H., Wineinger, M.A., Servis, M.E. 2004. Poststroke neuropsychiatric illness: an integrated approach to diagnosis and management. Curr Treat Options Neurol 6(5): 403-420. \r\nGlamcevski, M., Tan, C.T. 2000. Prevalence of post – stroke depression , a Malaysian study. Neurol Jo Southeast Asia 5: 51-3.\r\nHouse, A., Dennis, M., Moridge, L., Warlow, C., Hawton, K.,  Jones, L. 1991. Mood disorders in the first year after stroke. Br J Psychiatry 158: 83-92.\r\n Lai, S.M., Studenski, S., Duncan, P.W., Perera, S. 2002. Persisting consequences of stroke measured by the stroke impact scale. Stroke 33(7): 1840-4.  \r\nMackay, J., Mensah, G.A. 2004. Death from stroke. The Atlas of Heart Disease and Stroke, World Health Organization, Geneva, Switzerland, http://www.who.int/cardiovascular diseases/en/cvd_atlas_16_death_from_stroke.pdf [10 May 2008]  \r\nMukhtar, F., Oei, T.P.S. 2007. Depression in Malaysia: A comprehensive literature Review. Thesis dissertation for  PhD. Australia: University of Queensland.\r\nWilliams, L.S. 2005. Depression and stroke: cause or consequence? Semin Neurol 25(4): 396-409.\r\n'),
(6057, 6048, 'Ultrasound as a Triage Tool to Rule out Fractures', 'Nik Azlan Nik Muhamad, Department of Emergency Medicine, Faculty of Medicine,Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455702 Fax: +603-91456577 Email: nikazlanmuhamad@hotmail.com', 1707, 6034, 103, 111, 'Australian College for Emergency Medicine. 2013. Guidelines on the implementation of the Australasian Triage Scale in Emergency Department.  Melbourne: ACEM. http://www.bhi.nsw.gov.au [November 2013]\r\nBarata, I., Spencer, R., Suppiah, A., Raio, C., Ward, M.F., Sama, A. 2013. Integrated medical school ultrasound: development of an ultrasound vertical curriculum. Crit Ultrasound J  5(1): 6.\r\nBarata, I., Spencer, R., Suppiah, A., Raio, C., Ward, M.F., Sama, A. 2012. Emergency ultrasound in the detection of pediatric long-bone fractures. Pediatr Emerg Care 28(11): 1154-7.\r\nBitschnau, R., Gehmacher, O., Kopf, A., Scheier, M., Mathis, G. 1997. Ultrasound diagnosis of rib and sternum fractures. Ultraschall Med 18(4): 158-61.\r\nBlankstein, A. 2011. Ultrasound in the diagnosis of clinical orthopedics: The orthopedic stethoscope. World J Orthop 2(2): 13–24. \r\nBusse, J.W., Kaur, J., Mollon, B., Bhandari, M., Tornetta, P., Schünemann, H.J., Guyatt, G.H. 2009. Low intensity pulsed ultrasonography for fractures: systematic review of randomised controlled trials. BMJ 338: b351.\r\nChmiel, C., Huber, C.A., Rosemann, T., Zoller, M., Eichler, K., Sidler, P., Senn, O., 2011. Walk-ins seeking treatment at an emergency department or general  practitioner out-of-hours service: a cross-sectional comparison, BMC Health Serv Res 11: 94. \r\nColeman, P., Irons, R., Nicholl, J., 2001. Will alternative immediate care services reduce demands for non-urgent treatment at accident and emergency? Emerg Med J 18(6): 482-7.\r\nCooke, M.W., Arora, P., Mason, S. 2003. Discharge from triage: modelling the potential in different types of emergency department. Emerg Med J 20(2): 131–3.\r\nCross, K.P. 2011. Bedside ultrasound for pediatric long bone fractures. Clin Pediatr Emerg Med 12: 27-36.\r\nFenkl, R., von Garrel, T., Knaepler, H. 2012.Emergency diagnosis of sternum fracture with ultrasound. Unfallchirurg 95(8): 375-9.\r\nFriedrich, R.E., Heiland, M., Bartel-Friedrich, S. 2003. Potentials of ultrasound in the diagnosis of midfacial fractures. Clin Oral Investig 7(4): 226-9.\r\nHedelin, H.,   Goksör, L.A., Karlsson, J.,  Stjernström, S., 2013.  Ultrasound-assisted triage of ankle trauma can decrease the need for radiographic imaging. Am J Emerg Med 31(12): 1686–9.\r\nHeyworth, J. 2003. Ottawa ankle rules for the injured ankle. BMJ 326(7386): 405-6.\r\nJavadrashid, R., Khatoonabad, M., Shams, N., Esmaeili, F., Jabbari Khamnei, H. 2011. Comparison of ultrasonography with computed tomography in the diagnosis of nasal bone fractures. Dentomaxillofac Radiol 40(8): 486-91.\r\nKish, L. 1965. Survey Sampling. New York: John Wiley and Sons; 36.\r\nLaw, J., Macbeth, P.B. 2011. Ultrasound: from Earth to Space. Mcgill J Med 13(2): 59. \r\nLee, J.C., Healy, J. 2004. Sonography of Lower Limb Muscle Injury. AJR Am J Roentgenol 182(2): 341-51.\r\nLindelius, A., Törngren, S.,  Sondén, A.,  Pettersson, H., Adami, J. 2008.  Impact of surgeon-performed ultrasound on diagnosis of abdominal pain. Emerg Med J 25(8): 486–91. \r\nMarshburn, T.H., Legome, E., Sargsyan, A., Li, S.M., Noble, V.A., Dulchavsky, S.A., Sims, C., Robinson, D. 2004. Goal-directed ultrasound in the detection of long-bone fractures. J Trauma 57(2): 329–32.\r\nPian, L., Gillman, L.M., McBeth, P.B., Xiao, Z., Ball, C.G., Blaivas, M., Hamilton, D.R., Kirkpatrick, A.W. 2013. Potential use of remote telesonography as a transformational technology in underresourced and/or remote settings.  Emerg Med Int 2013: 986160. \r\nSippel, S., Muruganandan, K., Levine, A., Shah, S. 2011. Review article: Use of ultrasound in the developing world. Int  J Emerg Med 4: 72.\r\nSites, B.D., Antonakakis. J.G. 2009. Ultrasound guidance in regional anesthesia: state of the art review through challenging clinical scenarios. Local Reg Anesth 2: 1–14. \r\nStiell, I.G., Greenberg, G.H., McKnight, R.D., Nair, R.C., McDowell, I., Worthington, J.R. 1992. A study to develop clinical decision rules for the use of radiography in acute ankle injuries. Ann Emerg Med 21(4): 384–90.\r\nWarkentine, F.H., Horowitz, R., Pierce, M.C. 2014. The use of ultrasound to detect occult or unsuspected fractures in child abuse. Pediatr Emerg Care 30(1): 43-6. \r\nWeinberg, E.R., Tunik, M.G., Tsung, J.W. 2010. Accuracy of clinician-performed point-of-care ultrasound for the diagnosis of fractures in children and young adults. Injury. 41(8): 862-8.\r\n'),
(6059, 6050, 'The Estimation of Fetomaternal Haemorrhage by Flowcytometry', 'Dr. Nurasyikin Yusof, Department of Pathology, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455373 Fax: +603-91459483 Email: syikinby@gmail.com', NULL, 6034, 112, 122, 'Augustson, B.M., Fong, E.A., Grey, D.E., Davies, J.I., Erber, W.N. 2006. Postpartum anti-D: can we safely reduce the dose?. Med J Aust 184(12): 611-3.\r\nBayliss, K.M., Kueck, B.D., Johnson, S.T., Fueger, J.T., McFadden, P.W., Mikulski, D., Gottschall, J.L. 1991. Detecting fetomaternal haemorrhage: a comparison of five methods. Transfusion 31(4): 303-7.\r\nBCSH Blood Transfusion and General Haematology Task Force. 2011. Guidelines for the Estimation of Fetomaternal Haemorrhage. www.bcshguidelines.com. [Access date]\r\nBromilow, I.M., Duguid, J.K. 1997. Measurement of feto-maternal haemorrhage: a comparative study of three Kleihauer techniques and two flow cytometric methods. Clin Lab Haematol 19(2): 137-142.\r\nBrown, M., Wittwer, C. 2000. Flow cytometry: principles and clinical applications in haematology. Clin Chem 46(8 Pt 2): 1221-9.\r\nCorsetti, J.P., Cox, C., Leary, J.F., Cox, M.T., Blumberg, N., Doherty, R.A. 1987. Comparison of quantitative acid-elution technique and flow cytometry for detecting fetomaternal haemorrhage. Ann Clin Lab Sci 17(3):197-206.\r\nDavis, B.H., Olsen, S., Bigelow, N.C., Chen, J.C. 1998. Detection of fetal red cells in fetomaternal haemorrhage using a fetal haemoglobin monoclonal antibody by flow cytometry. Transfusion 38(8): 749-56.\r\nDuckett, J.R., Constantine, G. 1997. The Kleihauer technique: an accurate method of quantifying fetomaternal haemorrhage? Br J Obstet Gynaecol 104(7): 845-6.\r\nDuguid, J.K., Bromilow, I.M. 1999. Laboratory measurement of fetomaternal haemorrhage and its clinical relevance. Transfus Med Rev 13(1): 43-8. \r\nJanssen, W.C., Hoffmann, J.J. 2002. Evaluation of flow cytometric enumeration of foetal erythrocytes in maternal blood. Clin Lab Haematol 24(2): 89-92.\r\nJohnson, P.R., Hudson, C., Lee, D. 1993. The quantification of feto-maternal haemorrhage using flow cytometry. British Journal of Haematology 84(1): 48. \r\nJohnson, P.R., Tait, R.C., Austin, E.B., Shwe, K.H., Lee, D. 1995. Flow cytometry in diagnosis and management of large fetomaternal haemorrhage. J Clin Pathol 48(11): 1005-8.\r\nKennedy, G.A., Shaw, R., Just, S., Bryson, G., Battistutta, F., Rowell, J., Williams, B. 2003. Quantification of feto-maternal haemorrhage (FMH) by flow cytometry: anti-fetal haemoglobin labeling potential underestimates massive FMH in comparison to labeling with anti-D. Transfus Med 13(1): 25-33.\r\nKleihauer, E., Braun, H., Betke, K. 1957. Demonstration of fetal haemoglobin in erythrocytes by elution. Klin Wochenschr 35(12): 637-8.\r\nLloyd-Evans, P., Guest, A.R., Austin, E.B., Scott, M.L. 1999. Use of a phycoerythrin-conjugated anti-glycophorin A monoclonal antibody as a double label to improve the accuracy of FMH quantification by flow cytometry. Transfus Med 9(2): 155-160.\r\nMollison, P.L. 1972. Quantitation of transplacental haemorrhage. Br Med J 3(5818): 115.\r\nNance, S.J., Nelson, J.M., Arndt, P.A., Lam, H.C., Garratty, G.1989. Quantitation of fetal-maternal haemorrhage by flow-cytometry. Am J Clin Pathol 91(3): 288-92.\r\nNational Blood Transfusion Service Immunoglobulin Working Party. Recommendations for the use of anti-D immunoglobulin. Prescribers’ Journal 1991;31:137-45'),
(6061, 6052, 'Primary Low Grade Papillary Adenocarcinoma of Nasopharynx', 'Noor Dina Hashim, Department of Otorhinolaryngology-Head & Neck Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +60391456046 Fax: +60391456675 Email: dinahashim81@yahoo.com', 1154, 6034, 137, 140, 'Fu, C.H., Chang, K.P., Ueng, S.H., Wu, C.C., Hao, S.P. 2008. Primary thyroid-like papillary adenocarcinoma of the nasopharynx. Auris Nasus Larynx 35(4): 579-82.\r\nOhe, C., Sakaida, N., Tadokoro, C., Fukui, H., Asako, M., Tomoda, K., Uemure, Y. 2010. Thyroid-like low grade nasopharyngeal papillary adenocarcinoma: report of two cases. Pathol Int 60(2): 107-11.\r\nPineda-Daboin, K., Neto, A., Ochoa-Perez, V., Luna, M.A. 2006. Nasopharyngeal adenocarcinomas: a clinicopathologic study of 44 cases including immunohistochemical features of 18 papillary phenotypes. Ann Diagn Pathol 10(4): 215-21.\r\nWu, P.Y., Huang, C.C., Chen, H.K., Chien, C.Y. 2007. Adult thyroid-like low-grade nasopharyngeal papillary adenocarcinoma with thyroid transcription factor-1 expression. Otolaryngol Head Neck Surg 137(5): 837-8.\r\n'),
(6062, 6053, 'Homicide-Suicide: A Case Report', 'Ahmad Nabil Md Rosli, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455555 Fax: +603-91456681 Email: ahmadnabilrosli@gmail.com', 1293, 6034, 141, 145, 'Eliason, S. 2009. Murder-suicide: a review of the recent literature. J Am Acad Psychiatry Law 37(3): 371-6.\r\nHarper, D.W., Voigt, L. 2007. Homicide followed by suicide: An integrated theoretical perspective. Homicide Studies  11(4) : 295-318.\r\nLiem, M.C.A. 2010. Homicide followed by suicide: an empirical analysis. PhD Thesis. Utrecht University, the Netherlands.\r\nPanczak, R., Geissbuhler, M., Zwahlen, M., Killias, M., Tal, K., Egger, M. 2013. Homicide-suicides compared to homicides and suicides: Systematic review and meta-analysis. Forensic Sci Int 233(1): 28-36.\r\nRoma, P., Pazzelli, F., Pompili, M., Lester, D., Girardi, P., Ferracuti, S. 2012. Mental illness in homicide-suicide: a review. J Am Acad Psychiatry Law 40(4): 462-8.\r\nRosenbaum, M. 1990. The role of depression in couples involved in murder-suicide and homicide. Am J Psychiatry 147(8): 1036-9.\r\nFlynn, S.M., Swinson, N., While, D., Hunt, I.M., Roscoe, A., Rodway, C., Windfuhr, K., Kapur, N., Appleby, L., Shaw, J. 2009. Homicide followed by suicide: a cross-sectional study. J Forens Psychiatry Psychol 20(2): 306-21.\r\nZolkepli, F. 2013. Suicidal parents who killed their youngest daughter remanded until Sept 24. http://www.thestar.com.my/News/Nation/2013/09/19/Crime-Sucide-parents-remand/ [19 September 2013].\r\n'),
(6066, 6057, 'Charles Bonnet Syndrome', 'Rosdinom Razali, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456143 Fax: +603-91456681 Email: rosdinom@ppukm.ukm.edu.my', NULL, 6034, 146, 150, 'Arya, D.K. 1995. Charles Bonnet syndrome. Br J Psychiatry 167(1): 114-5.\r\nBassiony, M.M., Lyketsos, C.G. 2003. Delusions and hallucinations in Alzheimer’s disease: review of the brain decade. Psychosomatics 44(5): 388-401.\r\nCopeland, J.R., Dewey, M.E., Scott, A., Gilmore, C., Larkin, B.A., Cleave, N., McCracken, C.F., McKibbin, P.E. 1998. Schizophrenia and delusional disorder in older age: community prevalence, incidence, comorbidity, and outcome. Schizophr Bull 24(1): 153–61.\r\nFinucane, T.E. 2006. Neurontin for Charles Bonnet Syndrome. J Am Geriatric Soc 54(9): 1478. \r\nHäfner, H., Maurer, K., Löffler, W., an der Heiden, W., Munk-Jørgensen, P., Hambrecht, M., Riecher-Rössler, A. 1998. The ABC Schizophrenia Study: a preliminary overview of the results. Soc Psychiatry Psychiatr Epidemiol 33(8): 380–6.\r\nJacob, A., Prasad, S., Boggild, M., Chandratre, S. 2004. Charles-Bonnet syndrome-elderly people and visual hallucinations. BMJ 328(7455): 1552-4.\r\nMenon, G.J., Rahman, I., Menon, S.J., Dutton, G.N. 2003. Complex visual hallucinations in the visually impaired: the Charles Bonnet Syndrome. Surv Ophthalmol 48(1): 58-72. \r\nPliskin, N.H., Kiolbasa, T.A., Towle, V.L., Pankow, L., Ernest, J.T., Noronha, A., Luchins, D.J. 1996. Charles Bonnet syndrome: an early marker for dementia? J Am Geriatr Soc 44(9): 1055-61.\r\nRovner, B.W. 2006. The Charles Bonnet syndrome: a review of recent research. Curr Opin Ophthalmol 17(3): 275-7. \r\nShiraishi, Y., Terao, T., Ibi, K., Nakamura, J., Tawara, A. 2004. The rarity of Charles Bonnet syndrome. J Psychiatr Res 38(2): 207-13.\r\nWetherell, J.L., Jeste, D.V. 2004. Older adults with schizophrenia: patients are living longer and gaining researchers’ attention. Elder Care 3(2): 8-11. \r\n'),
(6073, 6064, 'PfNT3, Plasmodium falciparum Nucleoside permease', 'Dr. Pratima Kumari Sahu, Biochemistry & Molecular Biology, S. C. B. Medical College, Cuttack-753007, Odisha, India. Tel: 91+671+2313377 Fax: 91+671+2313377 Email: sahupratima@rediffmail.com', 6033, 6034, 123, 136, 'Biot, C., Chibale, K. 2006. Novel approaches to antimalarial drug discovery. Infect Disord Drug Targets 6(2): 173-204.\r\nCarter, N.S., Ben Mamoun, C., Liu, W., Silva, E.O., Landfear, S.M., Goldberg, D.E., Ullman, B. 2000. Isolation and functional characterization of the PfNT1 nucleoside transporter gene from Plasmodium falciparum. J Biol Chem 275(14): 10683-10691.\r\nCunha-Rodrigues, M., Prudencio, M., Mota, M.M., Haas, W. 2006. Antimalarial drugs - host targets (re)visited. Biotechnol J 1(3): 321-327.\r\nDownie, M.J., El Bissati, K., Bobenchik, A.M., Lochlainn, L.N., Americk, A., Zufferey, R., Kirk, K., Mamoun, C.B. 2010. PfNT2, a permease of the equilibrative nucleoside transporter family in the endoplasmic reticulum of Plasmodium falciparum. J Biol Chem 285(27): 20827-20833.\r\nEl Bissati, K., Downie, M.J., Kim, S.K., Horowitz, M., Carter, N., Ullman, B. 2008. Genetic evidence for the essential role of PfNT1 in the transport and utilization of xanthine, guanine, guanosine and adenine by Plasmodium falciparum. Mol Biochem Parasitol 161(2): 130- 139.\r\nEl Bissati, K., Zufferey, R., Witola, W.H., Carter, N.S., Ullman, B., Ben Mamoun, C. 2006. The plasma membrane permease PfNT1 is essential for purine salvage in the human malaria parasite, Plasmodium falciparum. 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Global overviews of Malaria. Indian J Med Res 106: 39-52.\r\nKyte, J., Doolittle, R.F. 1982. A simple method for displaying the hydropathic character of a protein. J Mol Biol 157(1): 105-132.\r\nMartin, R.E., Henry, R.I., Abbey, J. L., Clements, J.D., Kirk, K. 2005. The ‘permeome’ of the malaria parasite, an overview of the membrane transport proteins of Plasmodium falciparum. Genome Biol 6(3): R26.\r\nNicolle, R., Ben, M.C., Nicola, C.S., Daniel E.G., Ullman, B. 2001. Localization of the Plasmodium falciparum PfNT1 Nucleoside Transporter to the Parasite Plasma Membrane. Membrane transport structure, function and biogenesis. J Biol Chem 276(44): 41099-41099.\r\nQuashie, N.B., Ranford-Cartwright, L.C., de Koning, H.P. 2010. 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(6087, 6078, 'Gastro-Peritoneal Fistula Post Sleeve Gastrectomy', 'Dr. Nik Ritza Kosai, Minimally Invasive, Upper Gastro-Intestinal & Bariatric Surgery Unit, Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-9145 6202 Fax: +603-9145 6684 Email: nikkosai@yahoo.co.uk', NULL, 6034, 159, 164, 'Abraham, A., Virdi, R.P., Rajan, D., Singh, J., Mustacchia, P., Iqbal, J., Rizvon, K. 2012a. Gastrobronchial fistula following laparoscopic sleeve gastrectomy. BMJ Case Rep 2012.pii: bcr2012006789.\r\nBurgos, A.M., Braghetto, I., Csendes, A., Maluenda, F., Korn, O., Yarmuch, J., Gutierrez, L. 2009. Gastric leak after laparoscopic-sleeve gastrectomy for obesity. Obes Surg 19(12): 1672-7.\r\nCampos, J.M., Siqueira, L.T., Meira, M.R., Ferraz, A.A., Ferraz, E.M., Guimarães, M.J. Gastrobronchial fistula as a rare complication of gastroplasty for obesity. J Bar Pneumol. 2007 July-Aug; 33(4): 475-9.\r\nCasella, G., Soricelli, E., Rizzello, M., Trentino, P., Fiocca, F., Fantini, A., Salvatori, F.M., Basso, N. 2009. Nonsurgical treatment of staple line leaks after laparoscopic sleeve gastrectomy. Obes Surg 19(7): 821-6.\r\nCsendes, A., Burdiles, P., Burgos, A.M., Maluenda, F., Diaz, J.C. 2005. Conservative management of anastomotic leaks after 557 open gastric bypasses. Obes Surg 15(9): 1252-6.\r\nCsendes, A., Braghetto, I., León, P., Burgos, A.M. 2010. Management of leaks after laparoscopic sleeve gastrectomy in patients with obesity. J Gastrointest Surg 14(9): 1343-8.\r\nInbar, R., Santo, E., Subchi, Ael-A., Korianski, J., Halperin, Z., Greenberg, R., Avital, S. 2011. Insertion of removable self-expanding metal stents as a treatment for postoperative leaks and perforations of the oesophagus and stomach. Isr Med Assoc J 13(4): 230-3.\r\nMárquez, M.F., Ayza, M.F., Lozano, R.B., Morales Mdel, M., Díez, J.M., Poujoulet, R.B. 2010. Gastric leak after laparoscopic sleeve gastrectomy. Obes Surg 20(9): 1306-11.\r\nSerra, C., Baltasar, A., Andreo, L., Pérez, N., Bou, R., Bengochea, M., Chisbert, J.J. 2007. Treatment of gastric leaks with coated self-expanding stents after sleeve gastrectomy. Obes Surg 7(7): 866-72.\r\nTan, J.T., Kariyawasam, S., Wijeratne, T., Chandraratna, H.S. 2010. Diagnosis and management of gastric leaks after laparoscopic sleeve gastrectomy for morbid obesity. Obes Surg 20(4): 403-9.'),
(7257, 7248, 'On the Occasion of 10th Anniversary', 'Srijit Das, Editor-in-Chief, Medicine & Health, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91458638 Fax: +603-91456634 Email: drsrijit@gmail.com', NULL, 7246, 1, 1, '-'),
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(7270, 7261, 'Factors Associated with Asthma Exacerbation Revisit', 'Nik Azlan Nik Muhamad, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455703 Fax: +603-91456577 Email: nikazlanmuhamad@hotmail.com', 6026, 7246, 22, 28, 'Anderson H.R., Gupta R., Kapetanakis V., Asher M.I., Clayton T., Robertson C.F., Strachan D.P. 2008. International correlations between indicators of prevalence, hospital admissions and mortality for asthma in children. Int J Epidemiol 37(3): 573-82.\r\nAnderson H.R., Gupta R., Strachan D.P., Limb E.S. 2007. 50 years of asthma: UK trends from 1955 to 2004. Thorax 62(1): 85-90.\r\nEmerman, C.L., Cydulka, R.K. 1995. Factors associated with relapse after emergency department treatment for acute asthma. Ann Emerg Med 26(1): 6–11.\r\nEmerman, C.L., Woodruff, P.G., Cydulka, R.K., Gibbs, M.A., Pollack, C.V., Camargo, C.A. 1999. Prospective multicenter study of relapse following treatment for acute asthma among adults presenting to the emergency department. MARC investigators. Multicenter Asthma Research Collaboration. Chest 115(4): 919–27.\r\nGlobal Initiative For Asthma (GINA). 2015. Global Strategy For Asthma Management And Prevention Updated 2015. [GINA Web site]http://www.ginasthma.org/documents/4 [14 January 2016]\r\nRowe, B.H., Villa-Roel, C., Sivilotti, M.L., Lang, E., Borgundvaag, B., Worster, A., Walker, A, Ross, S. 2008. Relapse After Emergency Department Discharge for Acute Asthma. Acad Emerg Med 15(8): 709–17.\r\nRusso, M.J., McConnochie, K.M., McBride, J.T., Szilagyi, P.G., Brooks, A.M., Roghmann, K.J. 1999. Increase in admission threshold explains stable asthma hospitalization rates. Pediatrics 104(3 Pt 1): 454-62.\r\nWalsh-Kelly, C.M., Kelly, K.J., Drendel, A.L., Grabowski, L., Kuhn, E.M. 2008. Emergency department revisits for pediatric acute asthma exacerbations: association of factors identified in an emergency department asthma tracking system. Pediatr Emerg Care 24(8): 505-510.\r\nYaacob, I., Omar, R., Mustafa, W.N. 1991. The outcome of patients with acute bronchial asthma presenting to the emergency room. Singapore Med J 32(3): 166-168.'),
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(7295, 7286, 'Patient-Controlled Analgesia (PCA) for Orthopaedic Patients', 'Ho Siew Eng, Division of Nursing, International Medical University, No 126, Jalan, JalilPerjasa 19, Bukit Jalil, 57000 Kuala Lumpur, Malaysia. Tel: +603-86567228 Fax: +603-86567229 Email: hosieweng@imu.edu.my', 7023, 7246, 62, 71, 'Chumbley, G. Mountford, L. 2010. Patient-controlled analgesia infusion pumps for adults. Nurs Stand 25(8): 35-40.\r\nDihle, A., Bjolseth, G., Helseth, S. 2006. The gap between saying and doing in postoperative pain management. J Clin Nurs 15(4): 469–79.\r\nGuo, P., East, L., Arthur, A. 2012. A preoperative education intervention to reduce anxiety and improve recovery among Chinese cardiac patients: a randomized controlled trial. Int J Nurs Stud 49(2): 129-37.\r\nHerr, K., Coyne, PJ., McCaffery, M,, Manworren, R,, Merkel, S. 2011. Pain assessment in the patient unable to self-report: position statement with clinical practice recommendations. Pain Manag Nurs 12(4): 230-50.\r\nHo, S.E., Choy, Y.C., Rozainee, A. 2009. Survey of nurses’ knowledge and attitudes regarding post operative pain management at a teaching hospital in Malaysia. Medicine & Health 4(1): 47-52.\r\nHo, S.E., Wan Ahmad, L., Christopher, H.C.K., Tan, Z.Y., Nursharifah, M.S., Jaafar, M.Z., Choy, Y,C., Sabarul, A.M., Sharaf, I. 2015. The impact of a patient education package on outcomes of pain management following orthopedic surgery in a tertiary hospital in Malaysia. Medicine & Health 10(1): 58-65.\r\nHong, S.J., Lee, E. 2012. Effects of a structured educational programme on patient-controlled analgesia (PCA) for gynaecological patients in South Korea. J Clin Nurs 21(23-24): 3546-55.\r\nHudcova, J., McNicol, E., Quah, C., Lau, J., Carr, D.B. 2006. Patient controlled opioid analgesia versus conventional opioid analgesia for postoperative pain. Cochrane Database Syst Rev (4): CD003348.\r\nLovell, M.R., Forder, P.M., Stockler, M.R., Butow, P., Briganti, E.M., Chye, R., Goldstein, D., Boyle, F.M. 2010. A randomized controlled trial of a standardized educational intervention for patients with cancer pain. J Pain Symptom Manage 40(1): 49-59.\r\nMcNicol, E.D., Ferguson, M.C., Hudcova, J. 2015. Patient controlled opioid analgesia versus non-patient controlled opioid analgesia for postoperative pain. Cochrane Database Syst Rev 6: CD003348.\r\nPalmer, P.P., Miller, R.D. 2010. Current and developing methods of patient-controlled analgesia. Anesthesiol Clin 28(4): 587-99.\r\nPark, J.H., Kwon, M.A., Koo, M.S., Kim, Y.I., Kim, S.I., Kim, S.C., Kang, J.G., Cho, H.S., Lee, B.D., Kim, G.S. 2006. The effects of the preoperative education about analgesia and patient-controlled analgesia (PCA) on the consumption of analgesics after operation. Korean J Anesthesiol 51(6): 715–9.\r\nRothaug, J., Zaslansky, R., Schwenkglenks, M., Komann, M., Allvin, R., Backström, R., Brill, S., Buchholz, I., Engel, C., Fletcher, D., Fodor, L., Funk, P., Gerbershagen, H.J., Gordon,D.B., Konrad, C., Kopf, A., Leykin, Y., Pogatzki-Zahn, E., Puig, M., Rawal, N., Taylor, R.S., Ullrich, K., Volk, T., Yahiaoui-Doktor, M., Meissner, W. 2013. Patients’ perception of postoperative pain management: validation of the International Pain Outcomes (IPO) questionnaire. J Pain 14(11): 1361-70.\r\nViscusi, E.R., Schechter, L.N. 2006. Patient-controlled analgesia: Finding a balance between cost and comfort. Am J Health Syst Pharm 63(8 Suppl 1): S3-13.'),
(7297, 7288, 'Linguistic Validation of HypoSRQ', 'Tong Seng Fah, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-9145 6117 Fax: +603-91459479 Email: tsf@ppukm.ukm.edu.my', 7236, 7246, 72, 82, 'Brod, M., Christensen, t., Thomsen, T.L., Bushnell, D.M. 2011. The impact of non-severe hypoglycemic events on work productivity and diabetes management. Value Health 14(5): 665-671.\r\nGeelhoed-Duijvestijn, P.H., Pedersen-Bjergaard, U., Weitgasser, R., Lahtela, J., Jensen, M.M., Ostenson, C.G. 2013. Effects of patient-reported non-severe hypoglycemia on healthcare resource use, work-time loss, and wellbeing in insulin-treated patients with diabetes in seven European countries. J Med Econ 16(12): 1453-1461.\r\nHambleton, R.K., Patsula, L., 1998. Adapting tests for use in multiple languages and cultures Social Indicators Research 45: 153–171.\r\nHealth Psychology Research Ltd, 2014. HypoSRQ: Hypoglycaemia Symptom Rating Questionnaire - 18 Questions. URL:http://www.healthpsychologyresearch.com/information/currently-available-translated-questionnaires/hyposrq-hypoglycaemia-symptom-rating. [Accessed May 11, 2016]. Henderson, J., Allen, K.V., Deary, I.J., Frier, B.M. 2003. Hypoglycaemia in insulin treated Type 2 diabetes: frequency, symptoms and impaired awareness. Diabet Med 20(12): 1016-1021.\r\nJaap, A.J., Jones, G.C., Mccrimmon, R.J., Deary, I.J., Frier, B.M. 1998. Perceived symptoms of hypoglycaemia in elderly type 2 diabetic patients treated with insulin. Diabet Med 15(5): 398-401.\r\nMafauzy, M. 2006. Diabetes control and complications in public hospitals in Malaysia. Med J Malaysia 61(4): 477-483.\r\nMarrett, E., Stargardt, T., Mavros, P., Alexander, C.M. 2009. Patient reported outcomes in a survey of patients treated with oral antihyperglycaemic medications: associations with hypoglycaemia and weight gain. Diabetes Obes Metab 11(12): 1138-1144.\r\nMastura, I., Mimi, O., Piterman, L., Teng, C.L., Wijesinha, S. 2007. Self-monitoring of blood glucose among diabetes patients attending government health clinics. Med J Malaysia 62(2): 147-151.\r\nMccoy, R.G., Van Houten, H.K., Ziegenfuss, J.Y., Shah, N.D., Wermers, R.A., Smith, S.A. 2012. Increased mortality of patients with diabetes reporting severe hypoglycemia. Diabetes Care 35(9): 1897-1901.\r\nMurata, G.H., Duckworth, W.C., Shah, J.H., Wendel, C.S., Mohler M.J., Hoffman, R.M. 2005. Hypoglycemia in stable, insulin-treated veterans with type 2 diabetes: a prospective study of 1662 episodes. J Diabetes Complications 19(1): 10-17.\r\nTaylor, M.D., 2012. Evaluation of a new hypoglycaemia symptom measure: the hypoglycaemia symptom rating questionnaire (HypoSRQ). PhD thesis. Royal Holloway, University of London.\r\nTaylor, M.D., Hans, T.S., Ward, H., McBride, J., Yap, I., Bradley. C. 2013. Evaluation of the hypoglycaemia symptom rating questionnaire (’HypoSRQ’) and its relationship with hypoglycaemic episodes measured using continuous glucose monitoring [Abstract O-65]. Diabetes Technology & Therapeutics 15(Supp 1): A23–A24.\r\nVexiau, P., Mavros, P., Krishnarajah, G., lyu, R., Yin, D. 2008. Hypoglycaemia in patients with type 2 diabetes treated with a combination of metformin and sulphonylurea therapy in France. Diabetes Obes Metab 10 (Suppl 1): 16-24.\r\nWeinger, K., Jacobson, A.M., Draelos, M.T., Finkelstein, D.M., Simonson, D.C. 1995. Blood glucose estimation and symptoms during hyperglycemia and hypoglycemia in patients with insulin-dependent diabetes mellitus. Am J Med 98(1): 22-31.\r\nWild, D., Grove, A., Martin, M., Eremenco, S., McElroy, S., Verjee Lorenz, A., Erikson, P. 2005. Principles of Good Practice for the Translation and Cultural Adaptation Process for Patient-Reported Outcomes (PRO) Measures: report of the ISPOR Task Force for Translation and Cultural Adaptation. Value Health 8(2): 94-104.'),
(7300, 7291, 'ECT in a Patient with Low BMI', 'Hatta Sidi, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456142 Fax: +603-91456681 Email: hattasidi@hotmail.com', NULL, 7246, 83, 86, 'Callari, A., Lattanzi, L., Bartolommei, N., Cosentino, L., Luchini, F., Medda, P., Mauri, M. 2012. Electroconvulsive therapy in a case of catatonia with severe somatic complications. Journal of Psychopathology 18:164–9.\r\nLamprecht, H.C., Ferrier, I.N., Swann, A.G. 2005. The use of ECT in depressive illness. In The ECT Handbook. 2nd edition. Edited by Scott, A.I.F. Great Britain: Royal College of Psychiatrists; 9-24.\r\nPassione, R. 2004. Italian psychiatry in an international context: Ugo Cerletti and the case of electroshock. Hist Psychiatry 15(57 Pt 1): 83-104.\r\nPopeo, D.M. 2009. Electroconvulsive therapy for depressive episodes: a brief review. Geriatrics 64(4): 9-12.\r\nPoutanen, O., Huuhka, K., Perko, K. 2009. Severe anorexia nervosa, co-occurring major depressive disorder and electroconvulsive therapy as maintenance treatment: a case report. Cases J 2: 9362.\r\nUK ECT Review Group. 2003. Efficacy and safety of electroconvulsive therapy in depressive disorders: a systematic review and meta-analysis. Lancet 361(9360): 799–808.'),
(7303, 7294, 'Vaginismus Treatment', 'Hatta Sidi, Department of Psychiatry, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456142 Fax: +603-91456681 Email: hattasidi@hotmail.com', NULL, 7246, 87, 91, 'American Psychiatric Association. 2000. Diagnostic and Statistical Manual of Mental Disorders: DSM-IV. 4th edition. Text Revised. American Psychiatric Association: Washington, DC.\r\nAnnon, JS. 1976. The PLISSIT Model: A Proposed Conceptual Scheme for the Behavioral Treatment of Sexual Problems. Journal of Sex Education and Therapy 2(2): 1-15.\r\nIshak, W.W., Tobia, G. 2012. DSM-5 changes in diagnostic criteria of sexual dysfunctions. Reprod Syst Sex Disord 2: 122.\r\nFrank, E., Anderson, C., Kupfer, D.J. 1976. Profiles of couples seeking sex therapy and marital therapy. Am J Psychiatry 133(5): 559–562.\r\nMcGuire, H., Hawton, K. 2003. Interventions for vaginismus. Cochrane Database Syst Rev (1): CD001760.\r\nReissing, E.D., Binik, Y.M,, Khalife, S., Cohen, D., Amsel, R. 2004. Vaginal spasm, pain, and behavior: an empirical investigation of the diagnosis of vaginismus. Arch Sex Behav 33(1): 5-17.'),
(7306, 7297, 'Stridor in Children', 'Dr. Mohd Hisham Bin Mohd Isa, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91455606 Fax: +603-91456577 Email: mohimois@yahoo.com', 7000, 7246, 92, 95, 'Agada, F.O., Bell, J., Knight, L. 2006. Subglottic cysts in children: A 10-year review. Int J Pediatr Otorhinolaryngol 70(8): 1485-8.\r\nAksoy, E.A., Elsürer, C., Serin, G.M., Unal, O.F. 2012. Evaluation of pediatric subglottic cysts. Int J Pediatr Otorhinolaryngol 76(2): 240-3.\r\nJohnson, L.B., Rutter, M.J., Shott, S.R., Cotton, R.T. 2005. Acquired subglottic cysts in preterm infants. J Otolaryngol 34(2): 75-8.\r\nLim, J., Hellier, W., Harcourt, J., Leighton, S., Albert, D. 2003. Subglottic cysts: the Great Ormond Street experience. Int J Pediatr Otorhinolaryngol 67(5): 461-5.\r\nRansom, E.R., Antunes, M.B., Smith, L.P., Jacobs, I.N. 2009. Microdebrider resection of acquired subglottic cysts: case series and review of the literature. Int J Pediatr Otorhinolaryngol 73(12): 1833-6.\r\nWigger, H.J., Tang, P. 1968. Fatal laryngeal obstruction by iatrogenic subglottic cyst. J Pediatr 72(6): 815-820.\r\nZawadzka-Glos, L., Frackiewicz, M., Brzewski, M., Biejat, A., Chmielik, M. 2009. Difficulties in diagnosis of laryngeal cysts in children. Int J Pediatr Otorhinolaryngol 73(12): 1729-1731.'),
(7309, 7300, 'Contact Dermatitis to Traditional Medication', 'Leelavathi Muthupalaniappen, Department of Family Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603 91459475 Fax: +603 91459479 Email: drleelaraj@gmail.com', NULL, 7246, 96, 100, 'Ale, I.S., Maibacht, H. A. 2010. Diagnostic Approach in Allergic and Irritant Contact Dermatitis. Expert Rev ClinImmunol 6(2):291-310.\r\nBourke, J., Coulson, I., English, J., British Association of Dermatologists. 2001. Guideline for care of contact dermatitis.Br J Dermatol 145(6):877-85.\r\nFoti, C., Conserva, A., Antelmi, A., Lospalluti, L., Angelini, G., 2003. Contact dermatitis from peppermint and menthol in a local action transcutaneous patch. Contact Dermatitis 49(6):312-3.\r\nNiggemann, B., Gruber, C. 2003. Side effects of complementary and alternative medicine.Allergy 58(8):707-16. Noiles, K., Pratt, M. 2010. Contact dermatitis to Vicks VapoRub. Dermatitis 21(3):167-9.\r\nPapafragkakis, C., Ona, M.A., Reddy, M., Anand, S. 2016. Acute Hepatitis after Ingestion of a Preparation of Chinese Skullcap and Black Catechu for Joint Pain. Case Reports Hepatol 2016:4356749.\r\nSen, P., Ho, M.S., Ng, S.K., Yosipovitch, G. 2010. Contact dermatitis: a common adverse reaction to topical traditional Chinese medicine. Int J Dermatol 49(11):1255-60.\r\nSiti, Z.M., Tahir, A., Farah, A.I., Fazlin, S.M., Sondi, S., Azman, A.H., Maimunah, A.H., Haniza, M.A., Siti Haslinda, M.D., Zulkarnain, A.K., Zakiah, I., Zaleha, W.C. 2009. Use of traditional and complementary medicine in Malaysia: A baseline study. Complement Ther Med 17(5-6):292-9.\r\nStevenson, O.E., Finch, T.M., 2003. Allergic contact dermatitis from rectified camphor oil in Earex® ear drops. Contact Dermatitis 49(1):51.\r\nWilkinson, S.M., Beck, M. H. 1994. Allergic contact dermatitis from menthol in peppermint. Contact Dermatitis 30(1):42-3.\r\nWong, H.C.G. 2002. Allergic contact dermatitis from topical Chinese herbal medicine and generalized urticaria and angioedema. BCMJ 4 (4):184-7.'),
(7313, 7304, 'An Innovative and Reproducible Replacement to the Surgical Sandbag', 'Hardip S Gendeh. Department of Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456202 Fax: 03-91456640 Email: hardip88@gmail.com', NULL, 7246, 101, 104, 'Collins S, Lehman DS, McDougall EM, Clayman RV, Landman J. 2012. AUA BLUS Handbook of Laparoscopic and Robotic Fundamentals. American Urological Association Education & Research, Inc. available at https://www.auanet.org/education/blus-handbook.cfm. [31st July 2015].\r\nKnight DJW, Mahajan RP. 2004. Patient positioning in anaesthesia. Contin Educ Anaesth Crit Care Pain 4(5): 160-163.\r\nRozet, I, Vavilala, M.S. 2007. Risks and benefits of patient positioning during neurosurgical care. Anesthesiol Clin 25(3): 631-653.\r\nWisner DH, Jacoby RC. 2004. Injuries to the neck. In ACS Surgery: Principle and Practice. https://cirugiatraumaponiente.files.wordpress.com/2010/07/cuello.pdf. [1 Jan 2016].'),
(67602, 67588, 'Gelam Honey and Its Therapeutic Properties', 'Zar Chi Thent. Anatomy Unit, Faculty of Medicine, Universiti Teknologi MARA, Jalan Hospital, 47000 Sungai Buloh, Selangor, Malaysia. Tel: 006-03-61267307 Fax: 006-03-61267607 E-mail: zarrchii@gmail.com', 29409, 67583, 105, 116, 'A-Rahaman, N.L., Chua, L.S., Sarmidi, M.R., Aziz, R. 2013. Physicochemical and radical scavenging activities of honey samples from Malaysia. Agricultural Sciences 4: 46-51. \r\nAljady, A.M., Kamarudin, M.Y., Jamal, A.M., Mohd Yassim, M.Y. 2000. Biochemical study on the efficacy of Malaysian honey on inflicted wounds: an animal model. Medical Journal of Islamic Acedemy of Science 13(3): 125-32.\r\nAmes, B.N., Shigenaga, M.K., Hagen, T.M. 1993. Oxidants, antioxidants, and the degenerative diseases of aging. Proc Natl Acad Sci U S A 90(17): 7915-22.\r\nBatumalaie, K., Qvist, R., Yusof, K.M., Ismail, I.S., Sekaran, S.D. 2013. The antioxidant effect of the Malaysian Gelam honey on pancreatic hamster cells cultured under hyperglycemic conditions. Clin Exp Med 14(2): 185-95.\r\nBetteridge, D.J. 2000. What is oxidative stress?.Metabolism 49(2): 3-8. \r\nBogdanov, S. 1997. Nature and origin of the antibacterial substances in honey. LWT - Food Science and Technology 30(7): 748-53.\r\nBogdanov, S., Jurendic, T., Sieber, R., Gallmann, P. 2008. Honey for nutrition and health. J Am Coll Nutr 27(6): 677-89.\r\nBurkill, I.H. 1993. A Dictionary of the economic products of the Malay Pennisula.3rd Printing Publication unit, Ministry of Agriculture, Malaysia, Kuala Lumpur; 1: 1241-2444.\r\nChua, L.S., Rahaman, N.L., Adnan, N.A., Eddie Tan, T.T. 2013. Antioxidant activity of three honey samples in relation with their biochemical components. J Anal Methods Chem 2013: 313798.\r\nChua, L.S., Adnan, N.A., Abdul-Rahaman, N.L., Sarmidi, M.R., 2014. 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Gelam Honey Inhibits the Production of Proinflammatory, Mediators NO,PGE2, TNF-α, and IL-6 in Carrageenan-Induced Acute Paw Edema in Rats. Evid Based Complement Alternat Med 2012: 109636.\r\nHussein, S.Z., Yusoff, K.M., Makpol, S., Yusof, Y.A. 2011. Antioxidant capacities and total phenolic contents increase with gamma irradiation in two types of Malaysian honey. Molecules 16(8): 6378–95.\r\nHussein, S.Z., Mohd Yusoff, K., Makpol, S., Mohd Yusof, Y.A 2013. Gelam honey attenuates carrageenan-induced rat paw inflammation via NF-κB pathway. PLoS One 8(8): e72365.\r\nHussein, S.Z., Yusoff, K.M., Makpol, S., Mohd Yusof, Y.A. 2014. Does gamma irradiation affect physicochemical properties of honey? Clin Ter 165(2): e125-33. \r\nIwalewa, E.O., McGaw, L.J., Naidoo, V., Eloff, J.N. 2007. Inflammation: the foundation of diseases and disorders. A review of phytomedicines of South African origin used to treat pain and inflammatory conditions. 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Infection 26(1): 26-31. \r\nWen, C.T., Hussein, S.Z., Abdullah, S., Karim, N.A., Makpol, S., Mohd Yusof, Y.A. 2012. Gelam and Nenas honeys inhibit proliferation of HT 29 colon cancer cells by inducing DNA damage and apoptosis while suppressing inflammation. Asian Pac J Cancer Prev 13(4): 1605-1610.\r\nYusof, A.M., Abd Ghafar, N., Kamarudin, T.A., Hui, C.K., Yusof, Y.A. 2016. Gelam honey potentiates ex vivo corneal keratocytes proliferation with desirable phenotype expression. BMC Complement Altern Med 16:76.\r\n'),
(67605, 67591, 'Management of Fifth Metatarsal Fracture', 'Mohd Yusoff Yahaya, Orthopaedic Discipline, Surgical Science Cluster, Faculty of Medicine, Universiti Teknologi MARA, Sungai Buloh Campus, 47000 Selangor. Tel: +60361267314 Fax: +603-6126 5224 Email: yusoffyahaya@gmail.com', 29410, 67583, 131, 138, 'Akimau, P.I., Cawthron, K.L., Dakin, W.M., Chadwick, C., Blundell, C.M., Davies, M.B. 2016. Symptomatic treatment or cast immobilisation for avulsion fractures of the base of the fifth metatarsal. Bone Joint J 98(6): 806-11.\r\nAynardi, M., Pedowitz, D.I., Saffel, H., Piper, C., Raikin S.M. 2013. Outcome of nonoperative management of displaced oblique spiral fractures of the fifth metatarsal shaft. Foot Ankle Int 34(12): 1619-23.\r\nBishop, J.A., Braun, H.J., Hunt, K.J. 2015. Operative versus nonoperative treatment of jones fractures: A decision analysis model. Am J Orthop (Belle Mead NJ) 45(3): E69-76.\r\nChoi, J.H., Lee, K.T., Lee, Y.K., Lee, J.Y., Kim, H.R. 2013. Surgical results of zones I and II fifth metatarsal base fractures using hook plates. Orthopedics 36(1): e71-e74.\r\nDameron, T.B. 1975. Fractures and anatomic variations of the proximal portion of the fifth metatarsal. J Bone Joint Surg Am 57(6): 788-92.\r\nDe Lee, J.C. 1996.Fractures and dislocation of hip. In Rockwood and Green''s Fractures in Adults. 4th edition. Edited by Rockwood, C.A., Bucholz, R.W., Green, D.P, Heckman, J.D. Philadelphia: Lippincott-Raven; 1695-7.\r\nDeSandis, B., Murphy, C., Rosenbaum, A., Levitsky, M., O’Malley, Q., Konin, G., Drakos. M. 2016. Multiplanar CT analysis of fifth metatarsal morphology implications for operative management of zone II fractures. Foot Ankle Int 37(5): 528-36.\r\nFerguson, K.B., McGlynn, J., Kumar, C.S., Madeley, N.J., Rymaszewski. L. 2013. Fifth metatarsal fractures: is routine follow-up necessary? Injury 46(8): 1664-8.\r\nGranata, J.D., Berlet, G.C., Philbin, T.M., Jones, G., Kaeding, C.C., Peterson, K.S. 2015. 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J Foot Ankle Surg 53(5): 522-8.\r\nMahajan, V., Chung, H.W., Suh, J.S. 2011. Fractures of the proximal fifth metatarsal: percutaneous bicortical fixation. Clin Orthop Surg 3(2): 140-6.\r\nMarecek, G.S., Earhart, J.S., Croom, W.P., Merk, B.R. 2016. Treatment of Acute Jones Fractures Without Weightbearing Restriction. J Foot Ankle Surg 55(5): 961-4.\r\nMetzl, J., Olson, K., Davis, W.H., Jones, C., Cohen, B., Anderson, R. 2013. A clinical and radiographic comparison of two hardware systems used to treat Jones fracture of the fifth metatarsal. Foot Ankle Int 34(7): 956-61.\r\nNagao, M., Saita, Y., Kameda, S., Seto, H., Sadatsuki, R., Takazawa, Y., Yoshimura, M., Aoba, Y., Ikeda, H., Kaneko, K., Nozawa, M., Kim, S.G. 2012. Headless compression screw fixation of Jones fractures an outcomes study in Japanese athletes. Am J Sports Med 40(11): 2578-82.\r\nOchenjele, G., Ho, B., Switaj, P.J., Fuchs, D., Goyal, N., Kadakia, A.R. 2015. 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(67610, 67596, 'Ankle Injuries', 'Norzana Abd Ghafar, Department of Anatomy, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-92898605 Fax: +603-91458607 E-mail: norzana@ukm.edu.my', 13141, 67583, 117, 130, NULL),
(67614, 67600, 'Medication Wastages and Its Disposal amongst Patients in SSB Hospital', 'Nurolaini Kifli. PAPRSB Institute of Health Sciences, Universiti Brunei Darussalam (UBD), Jalan Tungku Link, Gadong, BE1410, Brunei Darussalam Tel: +6732463001 ext 1210 Fax: +673 2461081 E-mail: Nurolaini.kifli@ubd.edu.bn', 53295, 67583, 139, 150, 'Abahussain, E.A., Ball, D.E., Matowe, W.C. 2006. Practice and opinion towards disposal of unused medication in Kuwait. Med Princ Pract 15(4): 352-7.\r\nBraund, R., Peake, B.M., Shieffelbien, L. 2009. Disposal practices for unused medications in New Zealand. Environ Int 35(6): 952-5.\r\nEkedahl, A., Wergeman, L., Rydberg, T. 2003. Unused drugs in Sweden measured by returns to pharmacies. J Soc Adm Pharm 20: 26-31.\r\nJames, T.H., Helms, M.L., Braund, R. 2009. Analysis of medications returned to community pharmacies. Ann Pharmacother 43(10): 1631-5.\r\nKiyingi, K.S., Lauwo, J.A. 1993. Drugs in the home: danger and waste. World Health Forum 14(4): 381-4.\r\nKusturica, M.P., Sabo, A., Tomic, Z., Horvat, O., Solak, Z. 2012. Storage and disposal of unused medications: knowledge, behavior, and attitudes among Serbian people. Int J Clin Pharm 34(4): 604-10.\r\nLangley, C., Marriott, J., Mackridge, A., Daniszewski, R. 2005. An analysis of returned medicines in primary care. Pharm World Sci 27(4): 296-9.\r\nMinistry of Health, Malaysia (MOH). 2013. Return Your Medicines Program. Malaysia. http://www.pharmacy.gov.my/v2/en/content/return-your-medicines-program.html. [14 November 2016] \r\nNaing, L., Winn, T., Rusli, B.N. (2006) Sample Size Calculator for Prevalence Studies. http://www.kck.usm.my/ppsg/stats_resources.htm [10 November 2016]\r\nPersson, M., Sabelstrom, E., Gunnarsson, B. 2009. Handling of unused prescription drugs--knowledge, behaviour and attitude among Swedish people. Environ Int 35(5): 771-4.\r\nSeehusen, D.A., Edwards, J. 2006. Patient practices and beliefs concerning disposal of medications. J Am Board Fam Med 19(6): 542-54.\r\nToh, M.R., Chew, L. 2016. Turning waste medicines to cost savings: A pilot study on the feasibility of medication recycling as a solution to drug wastage. Palliat Med pii: 0269216316639798.\r\nTrueman, P., Lowson, K., Blighe, A., Meszaros, A., Wright, D., Glanville, J., Taylor, D., Newbold, J., Bury, M., Barber, N., Jani, Y. 2010. Evaluation of the scale, causes and costs of waste medicines. London: York Health Economics Consortium and School of Pharmacy, University of London.\r\nWest, L.M., Diack, L., Cordina,	M., Stewart, D. 2015. Applying the Delphi technique to define “medication wastage.” Eur J Hosp Pharm 22(5): 274–9.'),
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INSERT INTO `content_type_article` (`vid`, `nid`, `field_deck_value`, `field_cor_add_value`, `field_mid_nid`, `field_issue_nid`, `field_article_no_value`, `field_page_value`, `field_reference_value`) VALUES
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(67654, 67640, 'Expression of p53 Protein in Women within the Premenopausal Age', 'Wan Mazlina Md Saad, Medical Laboratory Technology Lecturer, Faculty of Health Sciences, Universiti Teknologi MARA (UiTM), Puncak Alam Campus, 42300, Selangor, Malaysia. Tel: +603-32584311 Fax: +603-32584599 E-mail: wanmaz755@salam.uitm.edu.my', 7354, 67583, 189, 198, 'Agrawal, A.K., Jele, M., Rudnicki, J., Grzebieniak, Z., Zukrowski, P., Nienartowicz, E. 2008. Molecular markers (c-erbB-2, p53) in breast cancer. Folia Histochem Cytobiol 46(4): 449-55.\r\nAmerican Cancer Society. 2015. Cancer Facts and Figures. Atlanta: American Cancer Society; 1-52.\r\nBartley, A.N., Ross, D.W. 2002. Validation of p53 immunohistochemistry as a prognostic factor in breast cancer in clinical practice. Arch Pathol Lab Med 126(4): 456-8.\r\nBergh, J., Norberg, T., Sjögren, S., Lindgren, A., Holmberg, L. 1995. Complete sequencing of the p53 gene provides prognostic information in breast cancer patients, particularly in relation to adjuvant systemic therapy and radiotherapy. Nat Med 1(10):1029-34.\r\nBertheau, P., Espié, M., Turpin, E., Lehmann, J., Plassa, L.F., Varna, M., Janin, A., de Thé, H. 2008. TP53 status and response to chemotherapy in breast cancer. Pathobiology 75(2): 132-9.\r\nBertheau, P., Lehmann-Che, J., Varna, M., Dumay, A., Poirot, B., Porcher, R., Turpin, E., Plassa, L.F., de Roquancourt, A., Bourstyn, E., de Cremoux, P., Janin, A., Giacchetti, S., Espié, M., de Thé, H. 2013. p53 in breast cancer subtypes and new insights into response to chemotherapy. Breast 22(Suppl 2): S27-S29. \r\nBloom, H. J., & Richardson, W. W. 1957. Histological grading and prognosis in breast cancer; a study of 1409 cases of which 359 have been followed for 15 years. Br J Cancer 11(3):359-77.\r\nCadoo, K.A., Traina, T.A., King, T.A. 2013. 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(67708, 67694, 'Prolong Placement of Tracheostomy Tube ', 'Marina Mat Baki, Department of Otorhinolaryngology-Head Neck Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456046 Fax: +603-91456675 E-mail: marinamatbaki@ppukm.ukm.edu.my', 29687, 67583, 298, 302, 'Backman, S., Bjorling, G., Johansson U.B., Lysdahl, M., Markstrom, A., Schedin, U., Aune, R.E., Frostell, C., Karlsson, S. 2009. Material wear of polymeric tracheostomy tubes: a six-month study. Laryngoscope 119(4): 657-64.\r\nEpstein, S. K. 2005. Late complications of tracheostomy. Respir Care 50(4): 542–9.\r\nSasaki, C.T., Horiuchi, M., Koss, N. 1979. Tracheostomy related subglottic stenosis: Bacteriologic pathogenesis. Laryngoscope 89(6 Pt 1): 857–65.\r\nSue, R.D., Susanto, I. 2003. Long term complications of artificial airways. Clin Chest Med 24(3): 457–71.\r\nStauffer, J.L., Olson, D.E., Petty, T.L. 1981. Complications and consequences of endotracheal intubation and tracheostomy. A prospective study of 150 critically ill adult patients. Am J Med 70(1): 65–76.\r\nYaremchuk, K. 2003. Regular tracheostomy tube changes to prevent formation of granulation tissue. Laryngoscope 113(1): 1-10.\r\n'),
(67715, 67701, 'CNPAS as a Rare Manifestation of Trisomy 8 Mosaicsm', 'Marina Mat Baki, Department of Otorhinolaryngology-Head Neck Surgery, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91456045 Fax: +603-91456675 E-mail: marinamatbaki@ppukm.ukm.edu.my', 29750, 67583, 303, 307, 'Agrawal, A., Agrawal, R. 2011. Warkany syndrome: a rare case report. Case Rep Pediatr 2011: 437101.  \r\nAl Abri, R., Javad, H,, Kumar, S., Bharga, D., Koul, R., Al Futaisi, A., Sankla, D. 2008. Congenital nasal pyriform aperture stenosis: first Case report in oman. Oman Med J 23(3): 192-4.\r\nArlis, H., Ward, R.F. 1992. Congenital nasal pyriform aperture stenosis: isolated abnormality vs developmental field defect. Arch Otolaryngol Head Neck Surg 118(9): 989-991. \r\nBelden, C.J., Mancuso, A.A., Schmalfuss, I.M. 1999. CT features of congenital nasal piriform aperture stenosis: initial experience Radiology 213(2): 495-501.\r\nGuilmin-Crépon, S., Garel, C., Baumann, C., Brémond-Gignac, D., Bailleul-Forestier, I., Magnier, S., Castanet, M., Czernichow, P., Van Den Abbeele, T., Léger, J. 2006. High proportion of pituitary abnormalities and other congenital defects in children with congenital nasal pyriform aperture stenosis. Pediatr Res 60(4): 478-84.\r\nLosken, A., Burstein, F.D., Williams, J.K., Turk, A.E. 2002. Congenital nasal pyriform aperture stenosis: diagnosis and treatment. Plast Reconstr Surg 109(5): 1506-11.\r\nRozner, L. 1964. Nasal obstruction due to restriction of the bony nasal inlet. Br J Plast Surg 17: 287-96.\r\nWiśniewska, M., Mazurek, M. 2002. Trisomy 8 mosaicism syndrome. J Appl Genet 43(1): 115-8.'),
(67717, 67703, 'Shake Till You Break', 'Dazlin Masdiana Sabardin, Department of Emergency Medicine, Faculty of Medicine, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-91458226 Fax: +60391456577 E-mail: dazlinms926@yahoo.com', 29795, 67583, 308, 312, 'Brackstone, M., Patterson, S.D., Kertesz, A.   2001.   Triple “E” syndrome: bilateral locked posterior fracture dislocation of the shoulders. Neurology 56(10): 1403-4.\r\nCagırmaz, T., Yapici, C., Orak, M.M., Guler, O. 2015. Bilateral femoral neck fractures after an epileptic attack: a case report.  Int J Surg Case Rep 6C: 107-10.\r\nCourt-Brown, C.M., Garg, A., Mcqueen, M.M. 2001. The epidemiology of proximal humeral fractures. Acta Orthop Scand  72(4): 365-71.\r\nFriedberg, R., Buras, J. 2005. Bilateral acetabular fractures associated with a seizure: a case report.  Ann Emerg Med  46(3): 260-2.\r\nGill, J.R., Murphy, C.G., Quansah, B., Carrothers, A.D. 2015. Seizure induced polytrauma; not just posterior dislocation of the shoulder. BMJ Case Rep 2015. pii:  bcr2015211445.\r\nGosens, T., Poels, P.J., Rondhuis, J.J. 2000. Posterior dislocation fractures of the shoulder in seizure disorders—two case reports and a review of literature.  Seizure  9(6): 446-8.\r\nKristiansen, B., Christensen, S. 1984. Fractures of the proximal end of the humerus caused by convulsive seizures. Injury 16(2): 108-9.\r\nLasanianos, N., Mouzopoulos, G. 2008. An undiagnosed bilateral anterior shoulder dislocation after a seizure: a case report. Cases J 1(1): 342.\r\nNoachtar, S. 1998. Bilateral fractures of the proximal humerus: a rare non-traumatic complication of generalized tonic-clonic seizures following withdrawal of antiepileptic medication.  J Neurol 245(2): 123-4.\r\nO’connor-Read, L., Bloch, B., Brownlow, H. 2007. A missed orthopaedic injury following a seizure: a case report.  J Med Case Rep 1: 20.\r\nRoux, A., Decroocq, L., El Batti, S., Bonnevialle, N., Moineau, G., Trojani, C., Boileau, P., de Peretti, F. 2012. Epidemiology of proximal humerus fractures managed in a trauma center.  Orthop Traumatol Surg Res 98(6): 715-9.\r\nWirrell, E.C. 2006. Epilepsy-related injuries. Epilepsia 47 (Suppl 1): 79-86.\r\n'),
(67723, 67709, 'CMV Retinitis in Dermatomyositis ', 'Christina Ng Wei Khee. Department of Ophthalmology, Hospital Selayang, Lebuhraya Selayang-Kepong, 68100 Batu Caves, Selangor, Malaysia/ Department of Ophthalmology, Universiti Kebangsaan Malaysia Medical Centre, Jalan Yaacob Latif, Bandar Tun Razak, 56000 Cheras, Kuala Lumpur, Malaysia. Tel: +603-61263333 Fax: +603-61371652 E-mail: christinangwk@gmail.com', 29954, 67583, 313, 318, 'Au Eong, K.G., Beatty, S., Charles, S.J. 1999. Cytomegalovirus retinitis in patients with acquired immune deficiency syndrome. Postgrad Med J 75(888): 585-90.\r\nDi Rollo, D., Abeni, D., Tracanna, M., Capo, A., Amerio, P. 2014. Cancer risk in dermatomyositis: a systematic review of the literature. G Ital Dermatol Venereol 149(5): 525-37.\r\nDownes, K.M., Tarasewicz, D., Weisberg, L.J., Cunningham, E.T. 2016. Good syndrome and other causes of cytomegalovirus retinitis in HIV-negative patients-case report and comprehensive review of the literature. J Ophthalmic Inflamm Infect 6(1): 3.\r\nHan, S.B., Lee, J.H., Lee, J.W., Chung, N.G., Cho, B., Kang, J.H., Kim, H.K., Lee, J.H., Lee, W.K. 2015. Cytomegalovirus retinitis diagnosed after completion of chemotherapy for acute lymphoblastic leukemia in an adolescent. J Pediatr Hematol Oncol 37(2): e128-e130.\r\nKim, H.R., Kim, S.D., Kim, S.H., Yoon, C.H., Lee, S.H., Park, S.H., Kim, H.Y. 2007. Cytomegalovirus retinitis in a patient with dermatomyositis. Clin Rheumatol 26(5): 801-3.\r\nKu, S.C., Yu, C.J., Chang, Y.L., Yang, P.C. 1999. Disseminated cytomegalovirus disease in a patient with systemic lupus erythematosus not undergoing immunosuppressive therapy. J Formos Med Assoc 98(12): 855-8.\r\nLangner-Wegscheider, B.J., ten Dam-van Loon, N., Mura, M., Faridpooya, K., de Smet, M.D. 2010. Intravitreal ganciclovir in the management of non-AIDS-related human cytomegalovirus retinitis. Can J Ophthalmol  45(2): 157-60.\r\nMori, T., Kameda, H., Ogawa, H., Iizuka, A., Sekiguchi, N., Takei, H., Nagasawa, H., Tokuhira, M., Tanaka, T., Saito, Y., Amano, K., Abe, T., Takeuchi, T. 2004. Incidence of cytomegalovirus reactivation in patients with inflammatory connective tissue diseases who are under immunosuppressive therapy. J Rheumatol 31(7): 1349-51.\r\nOlazagasti, J.M., Baez, P.J., Wetter, D.A., Ernste, F.C. 2015. Cancer risk in dermatomyositis: a meta-analysis of cohort studies. Am J Clin Dermatol 16(2): 89-98.\r\nYoshihara, S., Fukuma, N., Masago, R. 1999. Cytomegalovirus infection associated with immunosuppressive therapy in collagen vascular diseases. Ryumachi 39(5): 740-8.'),
(67727, 67713, 'Left Facial Tumour. What Is It? ', '-', NULL, 67583, 319, NULL, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_type_audio`
--

CREATE TABLE IF NOT EXISTS `content_type_audio` (
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  `field_op_audio_thumbnail_fid` int(11) DEFAULT NULL,
  `field_op_audio_thumbnail_list` tinyint(4) DEFAULT NULL,
  `field_op_audio_thumbnail_data` text,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_type_author`
--

CREATE TABLE IF NOT EXISTS `content_type_author` (
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  `field_author_photo_list` tinyint(4) DEFAULT NULL,
  `field_author_photo_data` text,
  `field_author_user_uid` int(10) unsigned DEFAULT NULL,
  `field_op_author_jobtitle_value` longtext,
  `field_institution_value` longtext,
  `field_email_author_email` varchar(255) DEFAULT NULL,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`),
  KEY `field_author_user_uid` (`field_author_user_uid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_type_author`
--

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(1367, 1358, NULL, NULL, NULL, NULL, 'Indocin | Purchase Cod', NULL, 'wrlhkzghe1979@yahoo.com'),
(1368, 1359, NULL, NULL, NULL, NULL, 'Minomycin | Buy Online Legal Issues', NULL, 'lszcpvt1985@hotmail.com'),
(1369, 1360, NULL, NULL, NULL, NULL, 'Sildalis | Order No Creditcard', NULL, 'dbzzblmw1988@hotmail.com'),
(1370, 1361, NULL, NULL, NULL, NULL, 'Himcolin | Buy Without Perscription At Alabama', NULL, 'vwrxahpxg1984@hotmail.com'),
(1371, 1362, NULL, NULL, NULL, NULL, 'Unisom | Can U Buy Online', NULL, 'umbubzcyu1976@yahoo.com'),
(1372, 1363, NULL, NULL, NULL, NULL, 'Provera |  Online Without A Prescription', NULL, 'ixnrlzq1980@yahoo.com'),
(1373, 1364, NULL, NULL, NULL, NULL, 'Pariet |  Bacon Hot Dogs Allergic Reaction', NULL, 'ongskjys1983@yahoo.com'),
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(1377, 1368, NULL, NULL, NULL, NULL, 'Nitroglycerin |  Without Rx Overnight Delivery No Rx', NULL, 'icxfbbov1984@gmail.com'),
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(1379, 1370, NULL, NULL, NULL, NULL, 'Allopurinol | Buy In Iowa', NULL, 'cogwmni1979@gmail.com'),
(1380, 1371, NULL, NULL, NULL, NULL, 'Skelaxin | Pediatric Dosing', NULL, 'gabcvnd1981@gmail.com'),
(1381, 1372, NULL, NULL, NULL, NULL, 'Bystolic | Online Us', NULL, 'rraklzw1979@hotmail.com'),
(1382, 1373, NULL, NULL, NULL, NULL, 'Cozaar | Canadian Without Prescription Online Doctors', NULL, 'uigcflib1988@yahoo.com'),
(1383, 1374, NULL, NULL, NULL, NULL, 'Luvox | Buy Without Prescriptin To Ship Overnight', NULL, 'jgwtjfpi1975@gmail.com'),
(1384, 1375, NULL, NULL, NULL, NULL, 'Alesse | Buy Saturday Delivery', NULL, 'uvkgsgc1983@yahoo.com'),
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(1386, 1377, NULL, NULL, NULL, NULL, 'Diclofenac |  Cheap', NULL, 'aisrnirnj1983@yahoo.com'),
(1387, 1378, NULL, NULL, NULL, NULL, 'Evista |  Separation Techniques', NULL, 'wddahmc1987@yahoo.com'),
(1388, 1379, NULL, NULL, NULL, NULL, 'Lotensin | To Buy In Al Phil Campbell', NULL, 'evpdjri1988@hotmail.com'),
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(1411, 1402, NULL, NULL, NULL, NULL, 'Zyban | Buy In Tijuana', NULL, 'chzyfqcem1985@hotmail.com'),
(1412, 1403, NULL, NULL, NULL, NULL, 'Zofran | Buy In The Uk', NULL, 'rbfdksda1984@yahoo.com'),
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(1415, 1406, NULL, NULL, NULL, NULL, 'Aldactone |  Online Order', NULL, 'nnadnyr1987@hotmail.com'),
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(1423, 1414, NULL, NULL, NULL, NULL, 'Cipro |  Online Uk', NULL, 'mblfmdisq1976@hotmail.com');
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(6095, 6086, NULL, NULL, NULL, NULL, 'Nasonex Nasal Spray | Buy Using Paypal', NULL, 'vzalfcr1980@yahoo.com'),
(6096, 6087, NULL, NULL, NULL, NULL, 'Cure Diabetes Naturally | Type 2 Diabetes Natural Treatment', NULL, 'zrusqseav1975@yahoo.com'),
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(6821, 6812, NULL, NULL, NULL, NULL, 'Sildenafil |  In Hypertension', NULL, 'phsscai1979@hotmail.com');
INSERT INTO `content_type_author` (`vid`, `nid`, `field_author_photo_fid`, `field_author_photo_list`, `field_author_photo_data`, `field_author_user_uid`, `field_op_author_jobtitle_value`, `field_institution_value`, `field_email_author_email`) VALUES
(6822, 6813, NULL, NULL, NULL, NULL, 'Triamterene |  Cost', NULL, 'lywpjxkn1979@hotmail.com'),
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(7316, 7307, NULL, NULL, NULL, NULL, 'Student', 'Faculty of Health Science, Universiti Kebangsaan Malaysia', 'bariahali@yahoo.com'),
(7317, 7308, NULL, NULL, NULL, NULL, 'master in radiology ', 'PPUKM', 'rayyanmardhiyah@gmail.com'),
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(7322, 7313, NULL, NULL, NULL, NULL, 'Purchase Eldepryl Depo-Eldepryl Billig', NULL, 'chcrxqgda1975@hotmail.com'),
(7323, 7314, NULL, NULL, NULL, NULL, 'Order Online Elimite Drug', NULL, 'eephdztfo1986@gmail.com'),
(7324, 7315, NULL, NULL, NULL, NULL, 'Order Emsam New Jersey', NULL, 'vjuawlw1976@yahoo.com'),
(7325, 7316, NULL, NULL, NULL, NULL, 'Senior Resident, Department of Laboratory Medicine, AIIMS, New Delhi-110029, India. ', 'AIIMS, New Delhi-', 'drgtudu@gmail.com'),
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(7327, 7318, NULL, NULL, NULL, NULL, 'Discounted Enalapril Order Arizona', NULL, 'vboxhpnl1977@hotmail.com'),
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(7353, 7344, NULL, NULL, NULL, NULL, 'Furosemid Without Prescription Medications', NULL, 'mpjfuxnz1986@yahoo.com'),
(7354, 7345, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Teknologi MARA', 'rashi353@salam.uitm.edu.my'),
(7355, 7346, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Teknologi MARA ', 'hairil@puncakalam.uitm.edu.my'),
(7356, 7347, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Teknologi MARA', 'wanmaz755@salam.uitm.edu.my'),
(7357, 7348, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Teknologi MARA', 'hairil@puncakalam.uitm.edu.my'),
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(7359, 7350, NULL, NULL, NULL, NULL, 'Senior Lecturer', 'Universiti Teknologi Mara ', 'hairil@puncakalam.uitm.edu.my'),
(7360, 7351, NULL, NULL, NULL, NULL, 'Senior Lecturer', 'Universiti Teknologi MARA', 'wanmaz755@salam.uitm.edu.my'),
(7361, 7352, NULL, NULL, NULL, NULL, 'Senior Lecturer', 'Universiti Teknologi MARA', 'wanmaz755@salam.uitm.edu.my'),
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(7464, 7455, NULL, NULL, NULL, NULL, NULL, NULL, 'kerpmicrachoop460@yahoo.com'),
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(7469, 7460, NULL, NULL, NULL, NULL, NULL, NULL, 'hosneurymen105@yahoo.com'),
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(7473, 7464, NULL, NULL, NULL, NULL, NULL, NULL, 'acovmaiclap514@yahoo.com'),
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(7475, 7466, NULL, NULL, NULL, NULL, NULL, NULL, 'specalimes152@yahoo.com'),
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(7480, 7471, NULL, NULL, NULL, NULL, NULL, NULL, 'hosneurymen105@yahoo.com'),
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(7482, 7473, NULL, NULL, NULL, NULL, NULL, NULL, 'acovmaiclap514@yahoo.com'),
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(8713, 8704, NULL, NULL, NULL, NULL, NULL, NULL, 'flooreswiper473@yahoo.com'),
(8714, 8705, NULL, NULL, NULL, NULL, NULL, NULL, 'tricofunin309@yahoo.com'),
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(25063, 25054, NULL, NULL, NULL, NULL, 'Pelé folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25064, 25055, NULL, NULL, NULL, NULL, 'Anyák napja folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25065, 25056, NULL, NULL, NULL, NULL, 'Évszakok folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25066, 25057, NULL, NULL, NULL, NULL, 'Zootropolis - Állati nagy balhé folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25067, 25058, NULL, NULL, NULL, NULL, 'Központi hírszerzés folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25068, 25059, NULL, NULL, NULL, NULL, 'A szerelem gyerekkel jön folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25069, 25060, NULL, NULL, NULL, NULL, 'Haifai kiköt? folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25070, 25061, NULL, NULL, NULL, NULL, 'Egy ?rült pillanat folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25071, 25062, NULL, NULL, NULL, NULL, 'Ha Isten úgy akarja folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25072, 25063, NULL, NULL, NULL, NULL, 'Kék bársony folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25073, 25064, NULL, NULL, NULL, NULL, 'Egy szó, mint száz: szerelem! folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25074, 25065, NULL, NULL, NULL, NULL, 'Elcserélt világ folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25075, 25066, NULL, NULL, NULL, NULL, 'Buy Now Voltaren-Gel Tabs', NULL, 'xocflcs1979@yahoo.com'),
(25076, 25067, NULL, NULL, NULL, NULL, 'A kommuna folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25077, 25068, NULL, NULL, NULL, NULL, 'Cloverfield Lane 10 folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25078, 25069, NULL, NULL, NULL, NULL, 'A F?nök folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25079, 25070, NULL, NULL, NULL, NULL, 'Robinson Crusoe folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25080, 25071, NULL, NULL, NULL, NULL, 'Kung Fu Panda 3 folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25081, 25072, NULL, NULL, NULL, NULL, 'Spotlight - Egy nyomozás részletei folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25082, 25073, NULL, NULL, NULL, NULL, 'Ifjúság folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25083, 25074, NULL, NULL, NULL, NULL, 'Miel?tt megismertelek folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25084, 25075, NULL, NULL, NULL, NULL, 'Izzie nyomában folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25085, 25076, NULL, NULL, NULL, NULL, 'A lánykirály folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25086, 25077, NULL, NULL, NULL, NULL, 'Ben Hur folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25087, 25078, NULL, NULL, NULL, NULL, 'Ratchet és Clank - A galaxis védelmez?i folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25088, 25079, NULL, NULL, NULL, NULL, 'Én és Kaminski folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25089, 25080, NULL, NULL, NULL, NULL, 'Madárkák folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25090, 25081, NULL, NULL, NULL, NULL, 'Bazi nagy görög lagzi 2. folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25091, 25082, NULL, NULL, NULL, NULL, 'Vad Kunság - A Puszta rejtett élete folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25092, 25083, NULL, NULL, NULL, NULL, 'Férfiak és csirkék folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25093, 25084, NULL, NULL, NULL, NULL, 'A szoba folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25094, 25085, NULL, NULL, NULL, NULL, 'Az én Szerelmem folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25095, 25086, NULL, NULL, NULL, NULL, '45 év folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25096, 25087, NULL, NULL, NULL, NULL, 'A lakás folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25097, 25088, NULL, NULL, NULL, NULL, 'A halott menyasszony folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25098, 25089, NULL, NULL, NULL, NULL, 'Magyar csapat - "...még 50 perc..." folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25099, 25090, NULL, NULL, NULL, NULL, 'Sarkköri ment?expedíció folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25100, 25091, NULL, NULL, NULL, NULL, 'A Vadász és a Jégkirályn? folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25101, 25092, NULL, NULL, NULL, NULL, 'Jöttünk, láttunk, visszamennénk 3. folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25102, 25093, NULL, NULL, NULL, NULL, 'Mama folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25103, 25094, NULL, NULL, NULL, NULL, 'Batman Superman ellen - Az igazság hajnala folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25104, 25095, NULL, NULL, NULL, NULL, 'A Beavatott-sorozat: A h?séges folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25105, 25096, NULL, NULL, NULL, NULL, 'A Keresztapa folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25106, 25097, NULL, NULL, NULL, NULL, 'Hétköznapi titkaink folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25107, 25098, NULL, NULL, NULL, NULL, 'Csokoládé folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25108, 25099, NULL, NULL, NULL, NULL, 'Mennyit ér egy ember? folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25109, 25100, NULL, NULL, NULL, NULL, 'Egyiptom istenei folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25110, 25101, NULL, NULL, NULL, NULL, 'A Zarándok - Paulo Coelho legjobb története folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25111, 25102, NULL, NULL, NULL, NULL, 'Deadpool folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25112, 25103, NULL, NULL, NULL, NULL, 'A boldogság titka folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25113, 25104, NULL, NULL, NULL, NULL, 'Alvin és a mókusok - A mókás menet folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25114, 25105, NULL, NULL, NULL, NULL, 'Lépcs?házi történetek folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25115, 25106, NULL, NULL, NULL, NULL, 'Truman folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25116, 25107, NULL, NULL, NULL, NULL, 'Mustang folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25117, 25108, NULL, NULL, NULL, NULL, 'Az él?halottak éjszakája folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25118, 25109, NULL, NULL, NULL, NULL, 'A kis herceg folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25119, 25110, NULL, NULL, NULL, NULL, 'Liza, a rókatündér folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25120, 25111, NULL, NULL, NULL, NULL, 'Legújabb testamentum folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25121, 25112, NULL, NULL, NULL, NULL, 'Csodapirula folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25122, 25113, NULL, NULL, NULL, NULL, 'Münó, a holdbéli manó folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25123, 25114, NULL, NULL, NULL, NULL, 'Szerelem Máltán folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25124, 25115, NULL, NULL, NULL, NULL, 'Éjfélkor folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25125, 25116, NULL, NULL, NULL, NULL, 'Szavak nélkül folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25126, 25117, NULL, NULL, NULL, NULL, 'Leviatán folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25127, 25118, NULL, NULL, NULL, NULL, 'Fehér tenyér folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25128, 25119, NULL, NULL, NULL, NULL, 'Külvárosi legenda folyam szabad magyar', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25129, 25120, NULL, NULL, NULL, NULL, 'Kochaj strumien darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25130, 25121, NULL, NULL, NULL, NULL, 'Zanim si? pojawi?e? strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25131, 25122, NULL, NULL, NULL, NULL, 'Angry Birds Film strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25132, 25123, NULL, NULL, NULL, NULL, 'Buying Ortho Tri-Cyclen No Prescription', NULL, 'dhgqvkjc1982@yahoo.com'),
(25133, 25124, NULL, NULL, NULL, NULL, 'Nice Guys. Równi go?cie strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25134, 25125, NULL, NULL, NULL, NULL, 'Warcraft: Pocz?tek strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25135, 25126, NULL, NULL, NULL, NULL, 'The Conjuring 2 strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25136, 25127, NULL, NULL, NULL, NULL, 'Wojownicze ?ó?wie ninja: Wyj?cie z cienia strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25137, 25128, NULL, NULL, NULL, NULL, 'Dzie? Bastylii strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25138, 25129, NULL, NULL, NULL, NULL, 'Zak?adnik z Wall Street strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25139, 25130, NULL, NULL, NULL, NULL, 'Idol z ulicy strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25140, 25131, NULL, NULL, NULL, NULL, 'Jak uratowa? mam? strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25141, 25132, NULL, NULL, NULL, NULL, 'X-Men: Apocalypse strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25142, 25133, NULL, NULL, NULL, NULL, 'Kapitan Ameryka: Wojna bohaterów strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25143, 25134, NULL, NULL, NULL, NULL, 'Ksi?ga d?ungli strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25144, 25135, NULL, NULL, NULL, NULL, 'Alicja po drugiej stronie lustra strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25145, 25136, NULL, NULL, NULL, NULL, 'Dzie? Matki strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25146, 25137, NULL, NULL, NULL, NULL, 'Zanim si? obudz? strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25147, 25138, NULL, NULL, NULL, NULL, 'S?siedzi 215 strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25148, 25139, NULL, NULL, NULL, NULL, 'Kung Fu Panda 3 strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25149, 25140, NULL, NULL, NULL, NULL, 'Mój przyjaciel orze? strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25150, 25141, NULL, NULL, NULL, NULL, 'Zwierzogród strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25151, 25142, NULL, NULL, NULL, NULL, 'Dzikie z natury strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25152, 25143, NULL, NULL, NULL, NULL, 'Ojciec strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25153, 25144, NULL, NULL, NULL, NULL, 'Szukaj?c Kelly strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25154, 25145, NULL, NULL, NULL, NULL, 'Wojna strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25155, 25146, NULL, NULL, NULL, NULL, 'Bóg w Krakowie strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25156, 25147, NULL, NULL, NULL, NULL, 'Baby Bump strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25157, 25148, NULL, NULL, NULL, NULL, 'Summer Camp strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25158, 25149, NULL, NULL, NULL, NULL, 'Opiekun strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25159, 25150, NULL, NULL, NULL, NULL, 'Dzielny kogut Maniek strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25160, 25151, NULL, NULL, NULL, NULL, 'Specjalny program strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25161, 25152, NULL, NULL, NULL, NULL, 'Pod tafl? oceanu strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25162, 25153, NULL, NULL, NULL, NULL, 'Blokada strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25163, 25154, NULL, NULL, NULL, NULL, 'Królowa strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25164, 25155, NULL, NULL, NULL, NULL, 'Cyrk w cyrku strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25165, 25156, NULL, NULL, NULL, NULL, 'Ostatni kurs strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25166, 25157, NULL, NULL, NULL, NULL, 'Skarb strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25167, 25158, NULL, NULL, NULL, NULL, 'How to Plan an Orgy in a Small Town strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25168, 25159, NULL, NULL, NULL, NULL, 'Lato Sangaile strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25169, 25160, NULL, NULL, NULL, NULL, 'Nienasyceni strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25170, 25161, NULL, NULL, NULL, NULL, 'By?y sobie cz?owieki strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25171, 25162, NULL, NULL, NULL, NULL, 'M?ody Mesjasz strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25172, 25163, NULL, NULL, NULL, NULL, 'Music Program strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25173, 25164, NULL, NULL, NULL, NULL, 'Pitbull. Nowe porzadki strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25174, 25165, NULL, NULL, NULL, NULL, 'Papusza strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25175, 25166, NULL, NULL, NULL, NULL, 'Afryka?ska przygoda 3D - safari nad Okavango darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25176, 25167, NULL, NULL, NULL, NULL, 'Królestwo zwierz?t strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25177, 25168, NULL, NULL, NULL, NULL, 'Playtime strumie? darmowe Polskie', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25178, 25169, NULL, NULL, NULL, NULL, 'Miles Ahead Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25179, 25170, NULL, NULL, NULL, NULL, 'Ninja Turtles Out of the Shadows (VO) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25180, 25171, NULL, NULL, NULL, NULL, 'Ninja Turtles 2 (VF) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25181, 25172, NULL, NULL, NULL, NULL, 'Camping 3 Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25182, 25173, NULL, NULL, NULL, NULL, 'ROH: Werther 2016 Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25183, 25174, NULL, NULL, NULL, NULL, 'Finding Dory Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25184, 25175, NULL, NULL, NULL, NULL, 'Bastille Day Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25185, 25176, NULL, NULL, NULL, NULL, 'A man called Ove Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25186, 25177, NULL, NULL, NULL, NULL, 'Tout de suite, maintenant Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25187, 25178, NULL, NULL, NULL, NULL, 'L''Outsider Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25188, 25179, NULL, NULL, NULL, NULL, 'Remember Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25189, 25180, NULL, NULL, NULL, NULL, 'Me Before You Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25190, 25181, NULL, NULL, NULL, NULL, 'Our Kind of Traitor Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25191, 25182, NULL, NULL, NULL, NULL, 'L''ideal Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25192, 25183, NULL, NULL, NULL, NULL, 'La Pazza Gioia (Folles de joie) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25193, 25184, NULL, NULL, NULL, NULL, 'Ma Loute Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25194, 25185, NULL, NULL, NULL, NULL, 'The Conjuring 2 Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25195, 25186, NULL, NULL, NULL, NULL, 'The Neon Demon Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25196, 25187, NULL, NULL, NULL, NULL, 'Warcraft: The Beginning (VA) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25197, 25188, NULL, NULL, NULL, NULL, 'Warcraft (VO) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25198, 25189, NULL, NULL, NULL, NULL, 'Money Monster Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25199, 25190, NULL, NULL, NULL, NULL, 'Ils sont partout Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25200, 25191, NULL, NULL, NULL, NULL, 'Retour chez ma mere Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25201, 25192, NULL, NULL, NULL, NULL, 'Alice im Wunderland 2: Hinter den Spiegeln Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25202, 25193, NULL, NULL, NULL, NULL, 'Alice Through the Looking Glass Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25203, 25194, NULL, NULL, NULL, NULL, 'Mother''s Day Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25204, 25195, NULL, NULL, NULL, NULL, 'Elle Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25205, 25196, NULL, NULL, NULL, NULL, 'X-Men: Apocalypse (VO) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25206, 25197, NULL, NULL, NULL, NULL, 'Julieta Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25207, 25198, NULL, NULL, NULL, NULL, 'The Nice Guys Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25208, 25199, NULL, NULL, NULL, NULL, 'The Angry Birds Movie Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25209, 25200, NULL, NULL, NULL, NULL, 'Cafe Society Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25210, 25201, NULL, NULL, NULL, NULL, 'Les malheurs de Sophie (Sophie''s Misfortunes) Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25211, 25202, NULL, NULL, NULL, NULL, 'Rico, Oskar und der Diebstahlstein Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25212, 25203, NULL, NULL, NULL, NULL, 'Captain America: Civil War (VO) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25213, 25204, NULL, NULL, NULL, NULL, 'Trumbo Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25214, 25205, NULL, NULL, NULL, NULL, 'Voices from Chernobyl (La Supplication) Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
(25215, 25206, NULL, NULL, NULL, NULL, 'Der Staat gegen Fritz Bauer Stream Gratis lëtzebuergesch', NULL, 'fwjwuhjf@guerrillamail.biz'),
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(67915, 67901, NULL, NULL, NULL, NULL, 'Zebeta | Purchase Side', NULL, 'kepnspzm1979@yahoo.com'),
(67916, 67902, NULL, NULL, NULL, NULL, 'Emsam | Buy Online', NULL, 'hmmsqib1977@gmail.com'),
(67917, 67903, NULL, NULL, NULL, NULL, 'Baclofen | Order Canada', NULL, 'estdrknk1976@yahoo.com'),
(67918, 67904, NULL, NULL, NULL, NULL, 'Baclofen | Buy Canada', NULL, 'pvxgbml1984@gmail.com'),
(67919, 67905, NULL, NULL, NULL, NULL, 'Hyzaar | Cheap Online', NULL, 'xrkyjll1981@yahoo.com'),
(67920, 67906, NULL, NULL, NULL, NULL, 'Zestoretic | Purchase Medication', NULL, 'ijjwowwd1988@yahoo.com'),
(67921, 67907, NULL, NULL, NULL, NULL, 'Atrovent | Buy Hfa Inhaler Online', NULL, 'hminsdshz1985@yahoo.com'),
(67922, 67908, NULL, NULL, NULL, NULL, 'Nitrofurantoin | Buy Uk', NULL, 'umuaizyal1979@yahoo.com'),
(67923, 67909, NULL, NULL, NULL, NULL, 'Luvox | Purchase Generic', NULL, 'xmqaous1983@gmail.com'),
(67924, 67910, NULL, NULL, NULL, NULL, 'Neurontin | Buy Canada', NULL, 'zccxqgmdx1985@yahoo.com'),
(67925, 67911, NULL, NULL, NULL, NULL, 'Lotrisone | Where To Buy', NULL, 'ixaauko1985@gmail.com'),
(67926, 67912, NULL, NULL, NULL, NULL, 'Serophene | Order Clomid Or', NULL, 'yoxfthha1985@yahoo.com'),
(67927, 67913, NULL, NULL, NULL, NULL, 'Amitriptyline | Buy Uk', NULL, 'zaquongd1978@gmail.com'),
(67928, 67914, NULL, NULL, NULL, NULL, 'Mircette | Buy', NULL, 'wfwqlmmfn1980@hotmail.com'),
(67929, 67915, NULL, NULL, NULL, NULL, 'Singulair | Where Can I Buy', NULL, 'tpecjhqc1982@gmail.com'),
(67930, 67916, NULL, NULL, NULL, NULL, 'Neurontin | Buy Overnight', NULL, 'txmgnflg1987@hotmail.com'),
(67931, 67917, NULL, NULL, NULL, NULL, 'Neurontin | Buy Medication', NULL, 'asiqbaxeb1984@gmail.com'),
(67932, 67918, NULL, NULL, NULL, NULL, 'Ventolin | Buy Nebulizer Solution', NULL, 'vvrxwbrv1985@gmail.com'),
(67933, 67919, NULL, NULL, NULL, NULL, 'Suhagra | Order', NULL, 'jkpuaba1988@gmail.com'),
(67934, 67920, NULL, NULL, NULL, NULL, 'Haldol | Buy Online', NULL, 'yzqbbte1976@hotmail.com'),
(67935, 67921, NULL, NULL, NULL, NULL, 'Baclofen | Buy Online', NULL, 'jlppfkxw1985@yahoo.com'),
(67936, 67922, NULL, NULL, NULL, NULL, 'Cytotec | Buy No Prescription England', NULL, 'yppacxch1977@hotmail.com'),
(67937, 67923, NULL, NULL, NULL, NULL, 'Eurax | Purchase', NULL, 'mlsliil1979@gmail.com'),
(67938, 67924, NULL, NULL, NULL, NULL, 'Prednisolone | Buy Steroids Uk', NULL, 'tsnwxkk1986@gmail.com'),
(67939, 67925, NULL, NULL, NULL, NULL, 'Norvasc | Order Cheap', NULL, 'cfgmaoig1982@hotmail.com'),
(67940, 67926, NULL, NULL, NULL, NULL, 'Propecia | Order Pills', NULL, 'erjmoump1983@hotmail.com'),
(67941, 67927, NULL, NULL, NULL, NULL, 'Seroquel |  Buy Canada', NULL, 'dquldfuut1986@hotmail.com'),
(67942, 67928, NULL, NULL, NULL, NULL, 'Amitriptyline |  Purchase Uk', NULL, 'gxclhyv1975@yahoo.com'),
(67943, 67929, NULL, NULL, NULL, NULL, 'Amitriptyline |  Buy Online Australia', NULL, 'qzenmmcz1977@yahoo.com'),
(67944, 67930, NULL, NULL, NULL, NULL, 'Lasix | Order Online Uk', NULL, 'ambifakz1986@gmail.com'),
(67945, 67931, NULL, NULL, NULL, NULL, 'Reglan | Purchase For Breastfeeding', NULL, 'dxewfzxk1978@yahoo.com'),
(67946, 67932, NULL, NULL, NULL, NULL, 'Vasotec | Purchase Medication', NULL, 'xoxfofr1983@gmail.com'),
(67947, 67933, NULL, NULL, NULL, NULL, 'Zoloft | Can You Order Online', NULL, 'tumfwgxq1981@gmail.com'),
(67948, 67934, NULL, NULL, NULL, NULL, 'Strattera | Buy Generic', NULL, 'avkrmti1981@yahoo.com'),
(67949, 67935, NULL, NULL, NULL, NULL, 'Omnicef | Buy', NULL, 'zbwlfjlgp1984@gmail.com'),
(67950, 67936, NULL, NULL, NULL, NULL, 'Omnicef | Purchase For Strep', NULL, 'tnuxmcd1977@gmail.com'),
(67951, 67937, NULL, NULL, NULL, NULL, 'Zestril | Purchase Prinivil', NULL, 'alnwywu1978@yahoo.com'),
(67952, 67938, NULL, NULL, NULL, NULL, 'Prednisone | Buy Canada', NULL, 'dsdveem1980@yahoo.com'),
(67953, 67939, NULL, NULL, NULL, NULL, 'Effexor | Cheap Xr 150', NULL, 'yvrpzkg1975@hotmail.com'),
(67954, 67940, NULL, NULL, NULL, NULL, 'Differin | Buy Cream Online Uk', NULL, 'bgpmwqipk1980@yahoo.com'),
(67955, 67941, NULL, NULL, NULL, NULL, 'Zofran | Can You Buy Otc', NULL, 'wdjtcgct1985@hotmail.com'),
(67956, 67942, NULL, NULL, NULL, NULL, 'Tadacip | Buy Usa', NULL, 'zvcopywkr1986@hotmail.com'),
(67957, 67943, NULL, NULL, NULL, NULL, 'Keflex | Buy For Dogs', NULL, 'sszkozows1978@gmail.com'),
(67958, 67944, NULL, NULL, NULL, NULL, 'Voltaren | How To Order', NULL, 'ibqptnatd1978@yahoo.com'),
(67959, 67945, NULL, NULL, NULL, NULL, 'Citalopram | Where Can I Buy', NULL, 'ksnhnvwvi1975@yahoo.com'),
(67960, 67946, NULL, NULL, NULL, NULL, 'Flomax | Buy Relief', NULL, 'sprtfmxim1981@hotmail.com'),
(67961, 67947, NULL, NULL, NULL, NULL, 'Nolvadex | Buy Online Reviews', NULL, 'xcfldiqog1977@hotmail.com'),
(67962, 67948, NULL, NULL, NULL, NULL, 'Naprosyn | Order Dosage', NULL, 'pkybbbr1988@hotmail.com'),
(67963, 67949, NULL, NULL, NULL, NULL, 'Famvir | Order', NULL, 'yqzyfeor1979@hotmail.com'),
(67964, 67950, NULL, NULL, NULL, NULL, 'Prandin | Purchase Medication', NULL, 'rtnkvgjun1983@hotmail.com'),
(67965, 67951, NULL, NULL, NULL, NULL, 'Bentyl | Buy Online', NULL, 'isnoybh1985@hotmail.com'),
(67966, 67952, NULL, NULL, NULL, NULL, 'Cephalexin | Buy 500Mg Without', NULL, 'nrmkfdu1984@yahoo.com'),
(67967, 67953, NULL, NULL, NULL, NULL, 'Valtrex | Cheap Generic Online', NULL, 'ptqiqbyr1983@gmail.com'),
(67968, 67954, NULL, NULL, NULL, NULL, 'Valtrex | Buy Without Insurance', NULL, 'iindxbjnh1981@yahoo.com'),
(67969, 67955, NULL, NULL, NULL, NULL, 'Eldepryl | Buy Online Uk', NULL, 'qgsrpef1988@yahoo.com'),
(67970, 67956, NULL, NULL, NULL, NULL, 'Mircette | Purchase And Weight', NULL, 'xpypodk1985@gmail.com'),
(67971, 67957, NULL, NULL, NULL, NULL, 'Proscar | Buy Merck', NULL, 'dwebcqcc1978@gmail.com'),
(67972, 67958, NULL, NULL, NULL, NULL, 'Estrace | Buy Cream From Canada', NULL, 'oyfcgheba1979@yahoo.com'),
(67973, 67959, NULL, NULL, NULL, NULL, 'Estrace | Order', NULL, 'prphtnesa1987@yahoo.com'),
(67974, 67960, NULL, NULL, NULL, NULL, 'Suhagra | Online Purchase Of', NULL, 'kvtvjaesj1978@hotmail.com'),
(67975, 67961, NULL, NULL, NULL, NULL, 'Serophene | Purchase For Men', NULL, 'ebinqlhsk1976@gmail.com'),
(67976, 67962, NULL, NULL, NULL, NULL, 'Robaxin | Order On-Line', NULL, 'lkvlohy1978@yahoo.com'),
(67977, 67963, NULL, NULL, NULL, NULL, 'Zocor | Order Lawsuits', NULL, 'vktoinjp1978@yahoo.com'),
(67978, 67964, NULL, NULL, NULL, NULL, 'Consultant Orthopaedic Surgeon and Lecturer', 'Universiti Kebangsaan Malaysia Medical Centre', 'ezeds007@yahoo.com.my'),
(67979, 67965, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia Medical Centre', 'ayesyah12@gmail.com'),
(67980, 67966, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia Medical Centre', 'nannyburn@yahoo.com'),
(67981, 67967, NULL, NULL, NULL, NULL, NULL, NULL, 'rohaizat_hassan@yahoo.com'),
(67982, 67968, NULL, NULL, NULL, NULL, 'Floxin |  Invisalign Cost With', NULL, 'qikmssch1987@yahoo.com'),
(67983, 67969, NULL, NULL, NULL, NULL, 'Baclofen | Order Online Uk', NULL, 'pikjlak1977@yahoo.com'),
(67985, 67971, NULL, NULL, NULL, NULL, 'Aricept | Cod Injection', NULL, 'mvsvovwac1979@hotmail.com'),
(67986, 67972, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'shegkar_10@hotmail.com'),
(67987, 67973, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'fitchemedic@gmail.com'),
(67988, 67974, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'iamhazab@gmail.com'),
(67989, 67975, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'ciksalmah_nani93@yahoo.com'),
(67990, 67976, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'raishanshariff@gmail.com'),
(67991, 67977, NULL, NULL, NULL, NULL, NULL, 'Universiti Kebangsaan Malaysia', 'gitu_3pb@yahoo.com'),
(67993, 67979, NULL, NULL, NULL, NULL, 'Aricept | Best Price Virginia', NULL, 'knzvqthwy1977@yahoo.com');
INSERT INTO `content_type_author` (`vid`, `nid`, `field_author_photo_fid`, `field_author_photo_list`, `field_author_photo_data`, `field_author_user_uid`, `field_op_author_jobtitle_value`, `field_institution_value`, `field_email_author_email`) VALUES
(67994, 67980, NULL, NULL, NULL, NULL, 'Diovan | Purchase 40Mg Magnesium', NULL, 'gsyvdgspo1976@gmail.com'),
(67995, 67981, NULL, NULL, NULL, NULL, 'Azulfidine | Cheap Western Union Canada', NULL, 'yonnbccop1982@yahoo.com'),
(67997, 67983, NULL, NULL, NULL, NULL, 'Sumycin | Get Overnight Delivery Visa', NULL, 'uexxiwidn1985@hotmail.com'),
(67998, 67984, NULL, NULL, NULL, NULL, 'Zocor | Cheap Sale Aalma', NULL, 'mnvnhcc1982@gmail.com'),
(67999, 67985, NULL, NULL, NULL, NULL, 'Coumadin | Cost Without Script Purchase', NULL, 'dacqbgql1975@hotmail.com'),
(68000, 67986, NULL, NULL, NULL, NULL, 'Midamor |  No Prescriptions Needed Cod', NULL, 'zqffywg1981@yahoo.com'),
(68001, 67987, NULL, NULL, NULL, NULL, 'Midamor | Order Dosage', NULL, 'ydenhtd1988@hotmail.com'),
(68002, 67988, NULL, NULL, NULL, NULL, 'Yasmin | Discount Uk Mastercard', NULL, 'zvqvavp1984@gmail.com'),
(68003, 67989, NULL, NULL, NULL, NULL, 'Cardizem | Overnight Buy', NULL, 'xqprlig1985@hotmail.com'),
(68004, 67990, NULL, NULL, NULL, NULL, 'Atrovent | Order Tablets', NULL, 'mvqnmtw1982@hotmail.com'),
(68005, 67991, NULL, NULL, NULL, NULL, 'Cozaar | Best 25 Mg Price', NULL, 'hadogvdr1975@hotmail.com'),
(68006, 67992, NULL, NULL, NULL, NULL, 'Differin | Price Drug', NULL, 'pdzxvpi1987@yahoo.com'),
(68007, 67993, NULL, NULL, NULL, NULL, 'Forzest | Purchase Price', NULL, 'jistran1978@hotmail.com'),
(68008, 67994, NULL, NULL, NULL, NULL, 'Abana |  Heartcare Overnight Disco', NULL, 'tbzwpfno1977@yahoo.com'),
(68009, 67995, NULL, NULL, NULL, NULL, 'Propecia | Pharmacy Order Generic', NULL, 'tppqvker1980@gmail.com'),
(68010, 67996, NULL, NULL, NULL, NULL, 'Buspar |  Cochabamba Comprar 42 Price', NULL, 'xykitqifk1980@yahoo.com'),
(68011, 67997, NULL, NULL, NULL, NULL, 'Plavix | Order 75Mg Online Now', NULL, 'ccrkjyeqr1988@gmail.com'),
(68012, 67998, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Generic Online Delivery', NULL, 'brtnxwtlv1985@yahoo.com'),
(68013, 67999, NULL, NULL, NULL, NULL, 'Naltrexone | Buy 3 Mg', NULL, 'yaekgqn1987@yahoo.com'),
(68014, 68000, NULL, NULL, NULL, NULL, 'Naltrexone |  Buying Store Online Sales', NULL, 'ddsureofx1988@yahoo.com'),
(68015, 68001, NULL, NULL, NULL, NULL, 'Naltrexone | Order Low Dose', NULL, 'iqkztozxc1984@gmail.com'),
(68016, 68002, NULL, NULL, NULL, NULL, 'Naltrexone | Purchase On Line Tabs', NULL, 'hppmgsxf1978@yahoo.com'),
(68017, 68003, NULL, NULL, NULL, NULL, 'Naltrexone | Order Online', NULL, 'fujhuetzr1975@yahoo.com'),
(68018, 68004, NULL, NULL, NULL, NULL, 'Naltrexone | Buy 50Mg Fedex Delivery', NULL, 'doahkenpa1985@hotmail.com'),
(68019, 68005, NULL, NULL, NULL, NULL, 'Naltrexone | Buy In Mexico', NULL, 'nwhzjemzv1985@yahoo.com'),
(68020, 68006, NULL, NULL, NULL, NULL, 'Propecia | Order Now Echeck', NULL, 'gzispzes1986@gmail.com'),
(68021, 68007, NULL, NULL, NULL, NULL, 'Dapoxetine | Cheap Vardenafil- Find No Rx', NULL, 'qkpcelukt1975@hotmail.com'),
(68022, 68008, NULL, NULL, NULL, NULL, 'Phenergan | Buy Online Tab Shop', NULL, 'zhorjxx1978@yahoo.com'),
(68023, 68009, NULL, NULL, NULL, NULL, 'Actos | Cheap Sales S46ia', NULL, 'bylosaolc1988@gmail.com'),
(68024, 68010, NULL, NULL, NULL, NULL, 'Serophene | To Buy Rx Purchase', NULL, 'kftnqsxx1977@hotmail.com'),
(68025, 68011, NULL, NULL, NULL, NULL, 'Hoodia | Otc 1200Mg Purchase', NULL, 'kilcvlewn1983@yahoo.com'),
(68026, 68012, NULL, NULL, NULL, NULL, 'Lopid |  Order Safe', NULL, 'vtnzslvt1985@yahoo.com'),
(68027, 68013, NULL, NULL, NULL, NULL, 'Z Code System |  Cost', NULL, 'pcmzysf1982@yahoo.com'),
(68029, 68015, NULL, NULL, NULL, NULL, 'Levlen | Buy Online Australia', NULL, 'sjticusd1978@hotmail.com'),
(68030, 68016, NULL, NULL, NULL, NULL, 'Cipralex | Can You Buy Online', NULL, 'hdzdckiv1985@yahoo.com'),
(68031, 68017, NULL, NULL, NULL, NULL, 'Grifulvin | Procerin Results', NULL, 'pjaojvr1987@yahoo.com'),
(68032, 68018, NULL, NULL, NULL, NULL, 'Neurontin | Baclofen Over The Counter Canada', NULL, 'mjjikyxzn1987@gmail.com'),
(68033, 68019, NULL, NULL, NULL, NULL, 'Tramadol |  Overnight Delivery Cheap', NULL, 'ljzxxlb1981@yahoo.com'),
(68034, 68020, NULL, NULL, NULL, NULL, NULL, NULL, 'ezzatyhusna@yahoo.com'),
(68035, 68021, NULL, NULL, NULL, NULL, NULL, NULL, 'izzati_06@yahoo.com.sg '),
(68036, 68022, NULL, NULL, NULL, NULL, NULL, NULL, 'ssuraiya@usm.my'),
(68038, 68024, NULL, NULL, NULL, NULL, 'Naltrexone |  With Canada Buy', NULL, 'mdinvxh1983@hotmail.com'),
(68039, 68025, NULL, NULL, NULL, NULL, 'Naltrexone | Cheap Consultation Online', NULL, 'tbiipjmfy1988@hotmail.com'),
(68040, 68026, NULL, NULL, NULL, NULL, 'Naltrexone |  Can I Buy Verkoop', NULL, 'rphkdzilv1979@gmail.com'),
(68041, 68027, NULL, NULL, NULL, NULL, 'Naltrexone | Buy France On Line', NULL, 'nnnogwd1980@hotmail.com'),
(68042, 68028, NULL, NULL, NULL, NULL, 'Naltrexone | Mail Order Without Rx', NULL, 'uafjcbkyq1976@hotmail.com'),
(68043, 68029, NULL, NULL, NULL, NULL, 'Medical Officer - postgraduate student', 'Universiti Kebangsaan Malaysia', 'nurule10@gmail.com'),
(68045, 68031, NULL, NULL, NULL, NULL, 'Cephalexin | Order 500', NULL, 'dfowmpdp1977@gmail.com'),
(68046, 68032, NULL, NULL, NULL, NULL, 'Topamax | Buy Canada', NULL, 'pciqjplp1984@yahoo.com'),
(68047, 68033, NULL, NULL, NULL, NULL, 'Tinidazole | Buy', NULL, 'sqvfnmtu1978@hotmail.com'),
(68048, 68034, NULL, NULL, NULL, NULL, 'Trimox | Buy', NULL, 'gmjtcfa1980@hotmail.com'),
(68049, 68035, NULL, NULL, NULL, NULL, 'Elavil | Buy Online Overnight', NULL, 'wpeduqzl1980@yahoo.com'),
(68050, 68036, NULL, NULL, NULL, NULL, 'Elavil | Buy Overnight Delivery', NULL, 'rncfbyaw1987@gmail.com'),
(68051, 68037, NULL, NULL, NULL, NULL, 'Ampicillin | Purchase Online', NULL, 'pgfmiho1987@gmail.com'),
(68052, 68038, NULL, NULL, NULL, NULL, 'Calan | Buy Calendars Online Canada', NULL, 'nkhrkbxcc1984@hotmail.com'),
(68053, 68039, NULL, NULL, NULL, NULL, 'Lisinopril | Buy 20 Mg Online', NULL, 'mihsajj1976@yahoo.com'),
(68054, 68040, NULL, NULL, NULL, NULL, 'Arcoxia | Purchase Prospect', NULL, 'owpgtfgyf1981@hotmail.com'),
(68055, 68041, NULL, NULL, NULL, NULL, 'Azulfidine | Order Manufacturer', NULL, 'vcrepauxf1975@hotmail.com'),
(68056, 68042, NULL, NULL, NULL, NULL, 'Doxazosin | Buy Online', NULL, 'ifshezi1984@hotmail.com'),
(68057, 68043, NULL, NULL, NULL, NULL, 'Doxazosin | Order Cardura', NULL, 'fiaqkmf1976@gmail.com'),
(68059, 68045, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Kebangsaan Malaysia', 'drkortho@gmail.com'),
(68060, 68046, NULL, NULL, NULL, NULL, 'Minomycin | Order Side', NULL, 'hfffqxvpz1975@hotmail.com'),
(68061, 68047, NULL, NULL, NULL, NULL, 'Prednisone | Buy For Cats', NULL, 'equixkk1975@hotmail.com'),
(68062, 68048, NULL, NULL, NULL, NULL, 'Tretinoin | Buy Retin A Cream', NULL, 'yftnwdkf1981@gmail.com'),
(68064, 68050, NULL, NULL, NULL, NULL, 'Famvir | Where To Buy', NULL, 'oftvwdoj1984@yahoo.com'),
(68065, 68051, NULL, NULL, NULL, NULL, 'Maxalt | Buy Online', NULL, 'tokzofksp1976@hotmail.com'),
(68066, 68052, NULL, NULL, NULL, NULL, 'Lorazepam | Buy Paypal', NULL, 'cibollrhl1983@gmail.com'),
(68067, 68053, NULL, NULL, NULL, NULL, 'Nizoral | Buy Cream', NULL, 'gltxrcerw1977@gmail.com'),
(68068, 68054, NULL, NULL, NULL, NULL, 'Clomipramine | Buy For Cats', NULL, 'upvsjin1979@yahoo.com'),
(68070, 68056, NULL, NULL, NULL, NULL, 'Ventolin | Buy London', NULL, 'tfnpiiu1982@yahoo.com'),
(68071, 68057, NULL, NULL, NULL, NULL, 'Paxil | Buy Generic', NULL, 'mnrlvri1984@gmail.com'),
(68072, 68058, NULL, NULL, NULL, NULL, 'Paxil | Buy Brand Name', NULL, 'selcqxf1987@gmail.com'),
(68073, 68059, NULL, NULL, NULL, NULL, 'Zestoretic | Purchase 20Mg', NULL, 'nygfcogz1978@gmail.com'),
(68074, 68060, NULL, NULL, NULL, NULL, 'Librium | Can You Order Online', NULL, 'ssiglxfm1979@yahoo.com'),
(68075, 68061, NULL, NULL, NULL, NULL, 'Tramadol | Order Pharmacy', NULL, 'pvrxmbw1985@yahoo.com'),
(68076, 68062, NULL, NULL, NULL, NULL, 'Zyban | Order Online', NULL, 'dsckyssw1981@hotmail.com'),
(68077, 68063, NULL, NULL, NULL, NULL, 'Lincocin | Buy Aqua', NULL, 'kqwbnydob1977@hotmail.com'),
(68078, 68064, NULL, NULL, NULL, NULL, 'Floxin | Purchase Antibiotic', NULL, 'egvhfxkij1980@hotmail.com'),
(68079, 68065, NULL, NULL, NULL, NULL, 'Citalopram | Buy 20Mg Tablets', NULL, 'pswancrsn1978@hotmail.com'),
(68080, 68066, NULL, NULL, NULL, NULL, 'Plavix | Purchase Online', NULL, 'xknuygu1977@hotmail.com'),
(68081, 68067, NULL, NULL, NULL, NULL, 'Seroquel |  To Buy Online', NULL, 'fjltncisa1981@hotmail.com'),
(68082, 68068, NULL, NULL, NULL, NULL, 'Naltrexone | Medication Buy', NULL, 'ekbxglx1987@yahoo.com'),
(68083, 68069, NULL, NULL, NULL, NULL, 'Naltrexone | Discount Without Insurance', NULL, 'allfcscn1986@yahoo.com'),
(68084, 68070, NULL, NULL, NULL, NULL, 'Naltrexone |  Order Now At Gloucestershire', NULL, 'aqdffhesi1975@yahoo.com'),
(68085, 68071, NULL, NULL, NULL, NULL, 'Naltrexone | Kaufen Verkoop Licensed Store', NULL, 'xpbfwnzhu1983@gmail.com'),
(68086, 68072, NULL, NULL, NULL, NULL, 'Naltrexone | Low Price Paypal', NULL, 'rhxjtpasv1979@hotmail.com'),
(68087, 68073, NULL, NULL, NULL, NULL, 'Naltrexone | Order Paypal', NULL, 'urrbxycs1981@hotmail.com'),
(68088, 68074, NULL, NULL, NULL, NULL, 'Naltrexone | Buy No Pr', NULL, 'hmbwwdxxo1986@gmail.com'),
(68089, 68075, NULL, NULL, NULL, NULL, 'Naltrexone | Shipped Ups Buy Samples', NULL, 'lpuukrkhn1978@hotmail.com'),
(68090, 68076, NULL, NULL, NULL, NULL, 'Naltrexone | Purchase-S American Express', NULL, 'ytzgctxxa1977@hotmail.com'),
(68091, 68077, NULL, NULL, NULL, NULL, 'Naltrexone | Prijzen On Line Cod', NULL, 'wzjsmuxto1982@gmail.com'),
(68092, 68078, NULL, NULL, NULL, NULL, 'Naltrexone | Buy 37 5 Online', NULL, 'ddjyltmwr1985@gmail.com'),
(68093, 68079, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Cod Amex Cost', NULL, 'cbmilstu1982@yahoo.com'),
(68094, 68080, NULL, NULL, NULL, NULL, 'Naltrexone | Discount Online', NULL, 'ybzyvqwta1976@yahoo.com'),
(68095, 68081, NULL, NULL, NULL, NULL, 'Naltrexone | Purchase Want Free Shipping', NULL, 'rsejbjk1983@hotmail.com'),
(68096, 68082, NULL, NULL, NULL, NULL, 'Naltrexone |  15Ml Best Buy', NULL, 'dingzlsj1987@yahoo.com'),
(68097, 68083, NULL, NULL, NULL, NULL, 'Naltrexone |  Ups Can I Buy', NULL, 'uilnpxb1987@hotmail.com'),
(68098, 68084, NULL, NULL, NULL, NULL, 'Naltrexone |  Money Order Visa', NULL, 'kkbffngt1975@yahoo.com'),
(68099, 68085, NULL, NULL, NULL, NULL, 'Naltrexone |  To Buy Worldwide', NULL, 'xgqedfffi1986@yahoo.com'),
(68100, 68086, NULL, NULL, NULL, NULL, 'Naltrexone | Best Buy Store', NULL, 'crrbzcp1979@gmail.com'),
(68101, 68087, NULL, NULL, NULL, NULL, 'Naltrexone | Discount Tablets  In Ripon', NULL, 'obgqqho1976@gmail.com'),
(68102, 68088, NULL, NULL, NULL, NULL, 'Armodafinil | Buy Nuvigil', NULL, 'urknhgezg1983@yahoo.com'),
(68103, 68089, NULL, NULL, NULL, NULL, 'Zolpidem | Order Without A Script', NULL, 'upszocij1984@yahoo.com'),
(68104, 68090, NULL, NULL, NULL, NULL, 'Keflex | Buy Online Canada', NULL, 'uyeyqfrjl1975@yahoo.com'),
(68105, 68091, NULL, NULL, NULL, NULL, 'Clomid | Buy', NULL, 'zllugks1979@yahoo.com'),
(68106, 68092, NULL, NULL, NULL, NULL, 'Minipress | Buyo', NULL, 'uefuxavs1975@hotmail.com'),
(68107, 68093, NULL, NULL, NULL, NULL, 'Keppra | Buy Australia', NULL, 'sewjioc1984@yahoo.com'),
(68108, 68094, NULL, NULL, NULL, NULL, 'Luvox | Purchase Medication', NULL, 'zglhiqwj1984@gmail.com'),
(68109, 68095, NULL, NULL, NULL, NULL, 'Propranolol | Buy 40Mg', NULL, 'xixvsqaxm1981@yahoo.com'),
(68110, 68096, NULL, NULL, NULL, NULL, 'Fluoxetine | Buy Prozac Online', NULL, 'bbwynfz1975@yahoo.com'),
(68111, 68097, NULL, NULL, NULL, NULL, 'Haldol | Order Decanoate', NULL, 'wyyiiklg1979@yahoo.com'),
(68112, 68098, NULL, NULL, NULL, NULL, 'Dhc |  Order Status', NULL, 'mmgzjqk1987@hotmail.com'),
(68113, 68099, NULL, NULL, NULL, NULL, 'Ditropan | Cod Legal Prices', NULL, 'gomngkw1978@gmail.com'),
(68114, 68100, NULL, NULL, NULL, NULL, 'Compazine | Buy Cod Saturday', NULL, 'gknmnojuu1978@hotmail.com'),
(68115, 68101, NULL, NULL, NULL, NULL, 'Imitrex | Buy Jcb', NULL, 'rtfwwdxm1977@hotmail.com'),
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(68326, 68312, NULL, NULL, NULL, NULL, 'Dapoxetine | Buy Real', NULL, 'lvsxnygdq1976@hotmail.com'),
(68327, 68313, NULL, NULL, NULL, NULL, 'Paroxetine | Buy', NULL, 'vobhyls1977@gmail.com'),
(68328, 68314, NULL, NULL, NULL, NULL, 'Aldactone | Buy', NULL, 'eqxifpu1979@gmail.com'),
(68329, 68315, NULL, NULL, NULL, NULL, 'Dihydrocodeine | Order Online Uk', NULL, 'ugzxeyvj1988@gmail.com'),
(68330, 68316, NULL, NULL, NULL, NULL, 'Phenergan | Buy Tablets Uk', NULL, 'syskacz1985@yahoo.com'),
(68331, 68317, NULL, NULL, NULL, NULL, 'Buspar | Buy On-Line', NULL, 'vkvxgil1977@yahoo.com'),
(68332, 68318, NULL, NULL, NULL, NULL, 'Furosemide | Buy Tablets', NULL, 'kljlpegv1976@hotmail.com'),
(68333, 68319, NULL, NULL, NULL, NULL, 'Fioricet | Order With Visa', NULL, 'bwgfohf1982@hotmail.com'),
(68334, 68320, NULL, NULL, NULL, NULL, 'Minocycline | Buy 100 Mg', NULL, 'ukqetrm1981@gmail.com'),
(68335, 68321, NULL, NULL, NULL, NULL, 'Naltrexone | Buy India', NULL, 'dsldzvl1979@yahoo.com'),
(68336, 68322, NULL, NULL, NULL, NULL, 'Naltrexone | Buy From India', NULL, 'ckfqlrnjk1987@yahoo.com'),
(68337, 68323, NULL, NULL, NULL, NULL, 'Naltrexone | Buy South Africa', NULL, 'jnkyomi1983@yahoo.com'),
(68338, 68324, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Injection', NULL, 'frtuzmuvy1986@hotmail.com'),
(68339, 68325, NULL, NULL, NULL, NULL, 'Seroflo | Order 500', NULL, 'nokuxmd1980@gmail.com'),
(68340, 68326, NULL, NULL, NULL, NULL, 'Lunesta | Purchase Online', NULL, 'ilfwrql1986@gmail.com'),
(68341, 68327, NULL, NULL, NULL, NULL, 'Dramamine | Where To Buy Patches', NULL, 'cwzvbvp1979@yahoo.com'),
(68342, 68328, NULL, NULL, NULL, NULL, 'Zetia | Buy In Canada', NULL, 'iwdiedwh1981@hotmail.com'),
(68343, 68329, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Uk', NULL, 'cbwjvaby1987@hotmail.com'),
(68344, 68330, NULL, NULL, NULL, NULL, 'Naltrexone | Order Revia', NULL, 'mdwqctsyo1976@yahoo.com'),
(68345, 68331, NULL, NULL, NULL, NULL, 'Naltrexone | Where To Purchase Online', NULL, 'dpgwiub1985@hotmail.com'),
(68346, 68332, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Implant', NULL, 'rsxfsfhqc1979@gmail.com'),
(68347, 68333, NULL, NULL, NULL, NULL, 'Naltrexone | Buy 50 Mg Uk', NULL, 'nnharhabr1986@yahoo.com'),
(68348, 68334, NULL, NULL, NULL, NULL, 'Naltrexone | Buy In Uk', NULL, 'gafmywjix1982@gmail.com'),
(68349, 68335, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Price', NULL, 'sqzvmezt1988@hotmail.com'),
(68350, 68336, NULL, NULL, NULL, NULL, 'Naltrexone | Where Can I Purchase', NULL, 'cwbweex1981@yahoo.com'),
(68351, 68337, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Tablets', NULL, 'jlfgefvl1979@hotmail.com'),
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(68355, 68341, NULL, NULL, NULL, NULL, 'Erythromycin | Buy Gel Online', NULL, 'nlobkje1988@hotmail.com'),
(68356, 68342, NULL, NULL, NULL, NULL, 'Chloramphenicol | Buy 1% Eye Ointment', NULL, 'boxkbdgd1978@hotmail.com'),
(68357, 68343, NULL, NULL, NULL, NULL, 'Benicar |  To Buy', NULL, 'uggwuvn1984@yahoo.com'),
(68358, 68344, NULL, NULL, NULL, NULL, 'Benoquin | Order Pills', NULL, 'xjzmzoj1975@gmail.com'),
(68359, 68345, NULL, NULL, NULL, NULL, 'Lynoral | Order Ingredients', NULL, 'fnfzjqvy1984@yahoo.com'),
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(68361, 68347, NULL, NULL, NULL, NULL, 'Topamax | Buy 25 Mg', NULL, 'inhpfae1987@hotmail.com'),
(68362, 68348, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Fedex Otc', NULL, 'yewhzktdu1988@gmail.com'),
(68363, 68349, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Spain', NULL, 'fqulzcgnr1985@gmail.com'),
(68364, 68350, NULL, NULL, NULL, NULL, 'Aldactone | Buy Cod Spirotone Fast', NULL, 'odvijwob1979@yahoo.com'),
(68365, 68351, NULL, NULL, NULL, NULL, 'Levothroid | Cheap Store', NULL, 'skppfwm1981@yahoo.com'),
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(68367, 68353, NULL, NULL, NULL, NULL, 'Clonidine | Overnight Find', NULL, 'htpanvl1986@yahoo.com'),
(68368, 68354, NULL, NULL, NULL, NULL, 'Dramamine | Cheap Generic No Prescription', NULL, 'edooghgqr1983@hotmail.com'),
(68369, 68355, NULL, NULL, NULL, NULL, 'Naltrexone |  Purchase Canada', NULL, 'sqcmjko1977@yahoo.com'),
(68370, 68356, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Revia', NULL, 'gxndacoxm1976@hotmail.com'),
(68371, 68357, NULL, NULL, NULL, NULL, NULL, NULL, 'drzuraidazmz@yahoo.com.my'),
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(68373, 68359, NULL, NULL, NULL, NULL, 'Naltrexone | Order Low Dose Online', NULL, 'xsymomqw1988@gmail.com'),
(68375, 68361, NULL, NULL, NULL, NULL, 'Naltrexone | Can I Buy Online', NULL, 'gmplwjnqk1985@gmail.com'),
(68376, 68362, NULL, NULL, NULL, NULL, 'Advair Diskus | Purchase', NULL, 'qedfxba1983@hotmail.com'),
(68377, 68363, NULL, NULL, NULL, NULL, 'Propranolol | Buy Paypal', NULL, 'hjouabt1980@yahoo.com'),
(68378, 68364, NULL, NULL, NULL, NULL, 'Pulmicort | Purchase Respules', NULL, 'yevbevkpr1980@gmail.com'),
(68379, 68365, NULL, NULL, NULL, NULL, 'Crestor | Buy 10 Mg', NULL, 'iuhugvjy1983@gmail.com'),
(68380, 68366, NULL, NULL, NULL, NULL, 'Artane | Order Generic Name', NULL, 'dtgvvkquf1983@gmail.com'),
(68381, 68367, NULL, NULL, NULL, NULL, 'Decadron | Buy', NULL, 'yuvzhayn1985@hotmail.com'),
(68382, 68368, NULL, NULL, NULL, NULL, 'Deltasone | Order Dosage', NULL, 'gzjcyohz1983@gmail.com'),
(68383, 68369, NULL, NULL, NULL, NULL, 'Paroxetine | Where To Buy Online', NULL, 'cutzuarra1981@gmail.com'),
(68384, 68370, NULL, NULL, NULL, NULL, 'Tofranil | Order', NULL, 'ncfkreazb1977@gmail.com'),
(68385, 68371, NULL, NULL, NULL, NULL, 'Tenormin | Buy Australia', NULL, 'orwripru1981@yahoo.com'),
(68386, 68372, NULL, NULL, NULL, NULL, 'Zocor | Cheap', NULL, 'snkcshpw1979@gmail.com'),
(68387, 68373, NULL, NULL, NULL, NULL, 'Flonase | Mail Order', NULL, 'xptdcnu1984@yahoo.com'),
(68388, 68374, NULL, NULL, NULL, NULL, 'Acticin | Order Permethrin', NULL, 'qrotfie1978@hotmail.com'),
(68389, 68375, NULL, NULL, NULL, NULL, 'Lioresal | Order Baclofen Canada', NULL, 'nltzqbcdt1976@gmail.com'),
(68390, 68376, NULL, NULL, NULL, NULL, 'Requip | Buy', NULL, 'vxqwnlfbk1987@gmail.com'),
(68391, 68377, NULL, NULL, NULL, NULL, 'Paxil | Order Cr Online', NULL, 'nmvxltoj1977@yahoo.com'),
(68392, 68378, NULL, NULL, NULL, NULL, 'Altace | Purchase Dosage', NULL, 'paleina1977@gmail.com'),
(68393, 68379, NULL, NULL, NULL, NULL, 'Tylenol | Buy 1 Online Canada', NULL, 'lwktszrw1976@hotmail.com'),
(68394, 68380, NULL, NULL, NULL, NULL, 'Orlistat | Where Can I Purchase', NULL, 'hqiidibk1982@yahoo.com'),
(68395, 68381, NULL, NULL, NULL, NULL, 'Diamox | Buy In Kathmandu', NULL, 'pawnofbmg1986@hotmail.com'),
(68396, 68382, NULL, NULL, NULL, NULL, 'Depakote | How To Order Levels', NULL, 'phruqbym1982@yahoo.com'),
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(68399, 68385, NULL, NULL, NULL, NULL, 'Lynoral | Purchase', NULL, 'xmethey1977@gmail.com'),
(68400, 68386, NULL, NULL, NULL, NULL, 'Tadacip | Order India', NULL, 'aceizhevm1979@hotmail.com'),
(68401, 68387, NULL, NULL, NULL, NULL, 'Minocycline | Buy Generic Online', NULL, 'caygbdbs1979@gmail.com'),
(68402, 68388, NULL, NULL, NULL, NULL, 'Zetia | Purchase In Canada', NULL, 'bhtcbrgqn1975@gmail.com'),
(68403, 68389, NULL, NULL, NULL, NULL, 'Keppra | Where To Buy 500Mg', NULL, 'cnoyjoqk1984@yahoo.com'),
(68404, 68390, NULL, NULL, NULL, NULL, 'Coreg | Buy Leads', NULL, 'jcjhjdj1983@hotmail.com'),
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(68406, 68392, NULL, NULL, NULL, NULL, 'Vasotec | Buy Online', NULL, 'ohqbjnh1980@yahoo.com'),
(68407, 68393, NULL, NULL, NULL, NULL, 'Zestril | Purchase Generic Name', NULL, 'ggbguyc1975@gmail.com'),
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(68410, 68396, NULL, NULL, NULL, NULL, 'Fosamax | Buy 10 Mg', NULL, 'dtjiuxi1982@hotmail.com'),
(68411, 68397, NULL, NULL, NULL, NULL, 'Bactrim | Order Ds', NULL, 'ltsrmcrmf1985@gmail.com'),
(68412, 68398, NULL, NULL, NULL, NULL, 'Metformin | Buy Online Usa', NULL, 'nkeefloc1976@hotmail.com'),
(68413, 68399, NULL, NULL, NULL, NULL, 'Metformin | Purchase 1000 Mg', NULL, 'sdhnymjg1986@gmail.com'),
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(68415, 68401, NULL, NULL, NULL, NULL, 'Rosuvastatin | Where Can I Buy', NULL, 'avgibcfnm1980@yahoo.com'),
(68416, 68402, NULL, NULL, NULL, NULL, 'Rosuvastatin |  Mail Order', NULL, 'zmbzloybp1985@hotmail.com'),
(68417, 68403, NULL, NULL, NULL, NULL, 'Florinef | Buy Online', NULL, 'gowcfmwt1988@yahoo.com'),
(68418, 68404, NULL, NULL, NULL, NULL, 'Promethazine | Order From Canada', NULL, 'tnzaxshn1985@gmail.com'),
(68419, 68405, NULL, NULL, NULL, NULL, 'Zebeta | Buy Generic', NULL, 'xthjlrd1985@hotmail.com'),
(68420, 68406, NULL, NULL, NULL, NULL, 'Carafate | Buy Australia', NULL, 'murwacjv1980@gmail.com'),
(68421, 68407, NULL, NULL, NULL, NULL, 'Midamor | Order', NULL, 'swpyxvyl1979@hotmail.com'),
(68422, 68408, NULL, NULL, NULL, NULL, 'Trimox | Purchase Side', NULL, 'aexddoyhy1979@gmail.com'),
(68423, 68409, NULL, NULL, NULL, NULL, 'Tricor | Order Samples', NULL, 'aqrvmldt1981@hotmail.com'),
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(68425, 68411, NULL, NULL, NULL, NULL, 'Oxazepam | Order Reviews', NULL, 'ozdcgmr1980@gmail.com'),
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(68428, 68414, NULL, NULL, NULL, NULL, 'Cafergot | Purchase By Novartis', NULL, 'lkznllhxr1979@hotmail.com'),
(68429, 68415, NULL, NULL, NULL, NULL, 'Acyclovir | Mail Order', NULL, 'ojouxqx1983@gmail.com'),
(68430, 68416, NULL, NULL, NULL, NULL, 'Nizoral | Buy Shampoo In Singapore', NULL, 'opqowudt1988@yahoo.com'),
(68431, 68417, NULL, NULL, NULL, NULL, 'Prinivil | Order Zestril', NULL, 'kxalazijl1987@yahoo.com'),
(68432, 68418, NULL, NULL, NULL, NULL, 'Chemical Pathologist, ', 'PPUKM', 'drdian@ppukm.ukm.edu.my'),
(68433, 68419, NULL, NULL, NULL, NULL, 'Medical officer', 'PPUKM', 'rabeya@ppukm.ukm.edu.my'),
(68434, 68420, NULL, NULL, NULL, NULL, 'haemotologist', 'PPUKM', 'suria.abdulaziz@gmail.com'),
(68435, 68421, NULL, NULL, NULL, NULL, 'Famvir | Buy Generic Online', NULL, 'eghzian1984@gmail.com'),
(68436, 68422, NULL, NULL, NULL, NULL, 'Prozac | Buy Uk', NULL, 'awsmrdlt1982@gmail.com'),
(68437, 68423, NULL, NULL, NULL, NULL, 'Hydrochlorothiazide | Buy Cheap', NULL, 'qtxmznej1977@gmail.com'),
(68438, 68424, NULL, NULL, NULL, NULL, 'Hydrochlorothiazide | Buy 12.5 Mg', NULL, 'hrgcdxfgr1979@hotmail.com'),
(68439, 68425, NULL, NULL, NULL, NULL, 'Zanaflex | Buy 4Mg On-Line', NULL, 'mbuemul1978@gmail.com'),
(68440, 68426, NULL, NULL, NULL, NULL, 'Micronase | Order Generic Name', NULL, 'fkcpmpcke1982@yahoo.com'),
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(68442, 68428, NULL, NULL, NULL, NULL, 'Maxalt | Buy Mlt Online', NULL, 'hpwaqwfk1987@gmail.com'),
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(68446, 68432, NULL, NULL, NULL, NULL, 'Tetracycline | Purchase Antibiotics Drugs', NULL, 'dwhaaekzk1981@yahoo.com'),
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(68448, 68434, NULL, NULL, NULL, NULL, 'Proscar | Buy Generic Online', NULL, 'hnbcayumh1982@gmail.com'),
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(68454, 68440, NULL, NULL, NULL, NULL, 'Furosemide | Buy For Dogs Online', NULL, 'bbuuukpuw1978@gmail.com'),
(68455, 68441, NULL, NULL, NULL, NULL, 'Naltrexone | Order Tablets', NULL, 'zbdkwkq1978@gmail.com'),
(68456, 68442, NULL, NULL, NULL, NULL, 'Proventil | Where To Buy Inhaler', NULL, 'jmfazsqmo1985@hotmail.com'),
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(68458, 68444, NULL, NULL, NULL, NULL, 'Glucophage | Buy Online In Usa', NULL, 'ygumonqcn1975@hotmail.com'),
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(68467, 68453, NULL, NULL, NULL, NULL, 'Paxil | Buy Uk', NULL, 'yiygrelo1975@gmail.com'),
(68468, 68454, NULL, NULL, NULL, NULL, 'Zithromax | Buy Online Fast Shipping', NULL, 'jgxavhen1981@yahoo.com'),
(68469, 68455, NULL, NULL, NULL, NULL, 'Diazepam | Buy 5Mg Uk', NULL, 'jygoqcwrd1982@hotmail.com'),
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(68475, 68461, NULL, NULL, NULL, NULL, 'Advair Diskus | Buy 250\\/50 On-Line', NULL, 'bsthdctg1985@yahoo.com'),
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(68478, 68464, NULL, NULL, NULL, NULL, 'Minocycline | Buy Canada', NULL, 'zewcrfrhv1976@gmail.com'),
(68479, 68465, NULL, NULL, NULL, NULL, 'Acyclovir | Buy In Mexico', NULL, 'hguhllpr1985@yahoo.com'),
(68480, 68466, NULL, NULL, NULL, NULL, 'Colospa | Order Retard', NULL, 'fxelwnxp1988@hotmail.com'),
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(68484, 68470, NULL, NULL, NULL, NULL, 'Stromectol | Buy Over The Counter', NULL, 'lulbvdvz1983@gmail.com'),
(68485, 68471, NULL, NULL, NULL, NULL, 'Phentermine | Buy 60 Mg', NULL, 'bjdysmwg1987@yahoo.com'),
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(68488, 68474, NULL, NULL, NULL, NULL, 'Azulfidine | Order 500', NULL, 'jbztwqgjs1985@yahoo.com'),
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(68490, 68476, NULL, NULL, NULL, NULL, 'Desyrel | Purchase Dosage', NULL, 'qypgnkui1981@yahoo.com'),
(68491, 68477, NULL, NULL, NULL, NULL, 'Lincocin | Buy Side', NULL, 'jmqmqyw1985@yahoo.com'),
(68492, 68478, NULL, NULL, NULL, NULL, 'Remeron | Buy Online Uk', NULL, 'aoedkzgpf1982@gmail.com'),
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(68495, 68481, NULL, NULL, NULL, NULL, 'Calan | Order Calendar 2017', NULL, 'lebkuhpix1979@hotmail.com'),
(68496, 68482, NULL, NULL, NULL, NULL, 'Cafergot | Buy Australia', NULL, 'rczltcs1983@yahoo.com'),
(68497, 68483, NULL, NULL, NULL, NULL, 'Zolpidem | Buy Cheap', NULL, 'mzzebomga1980@gmail.com'),
(68498, 68484, NULL, NULL, NULL, NULL, 'Tofranil | Buy Online', NULL, 'iharyrzc1975@gmail.com'),
(68499, 68485, NULL, NULL, NULL, NULL, 'Tadalis | Buy', NULL, 'mwpssorbr1980@hotmail.com'),
(68500, 68486, NULL, NULL, NULL, NULL, 'Lunesta | Buy From India', NULL, 'fxryggduz1985@gmail.com'),
(68501, 68487, NULL, NULL, NULL, NULL, 'Doxycycline | Cheap Tablets', NULL, 'cbledakpz1982@yahoo.com'),
(68502, 68488, NULL, NULL, NULL, NULL, 'Depakote | Cheap', NULL, 'pcrkuem1983@hotmail.com'),
(68503, 68489, NULL, NULL, NULL, NULL, 'Reglan | Buy', NULL, 'pkqwadr1975@yahoo.com'),
(68504, 68490, NULL, NULL, NULL, NULL, NULL, NULL, 'drpunitha_malar@yahoo.com'),
(68505, 68491, NULL, NULL, NULL, NULL, NULL, NULL, 'nanvsp@gmail.com'),
(68507, 68493, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Low-Dose Australia', NULL, 'lhbnsipw1978@yahoo.com'),
(68508, 68494, NULL, NULL, NULL, NULL, 'Elimite | Where To Buy Lotion', NULL, 'kfmozfwai1977@hotmail.com'),
(68509, 68495, NULL, NULL, NULL, NULL, 'Elimite | Buy Cream', NULL, 'ahnlcpb1981@hotmail.com'),
(68510, 68496, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Online Usa', NULL, 'bzvlngj1975@yahoo.com'),
(68511, 68497, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Online', NULL, 'qhugifyt1988@yahoo.com');
INSERT INTO `content_type_author` (`vid`, `nid`, `field_author_photo_fid`, `field_author_photo_list`, `field_author_photo_data`, `field_author_user_uid`, `field_op_author_jobtitle_value`, `field_institution_value`, `field_email_author_email`) VALUES
(68513, 68498, NULL, NULL, NULL, NULL, 'Naltrexone | Where To Purchase', NULL, 'qitnxss1988@yahoo.com'),
(68514, 68499, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Powder', NULL, 'leyjmjq1985@yahoo.com'),
(68515, 68500, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Thailand', NULL, 'ebopmoo1987@gmail.com'),
(68516, 68501, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Without A Script', NULL, 'youqeozog1977@yahoo.com'),
(68517, 68502, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Hydrochloride', NULL, 'iwnsxyrp1986@gmail.com'),
(68518, 68503, NULL, NULL, NULL, NULL, 'Naltrexone | How To Buy Online', NULL, 'epuhcgkp1982@hotmail.com'),
(68519, 68504, NULL, NULL, NULL, NULL, 'ประกันรถยนต์', NULL, 'EmmaBurkhartn760@hotmail.com'),
(68520, 68505, NULL, NULL, NULL, NULL, 'Dihydrocodeine | Buy Next Day Delivery', NULL, 'fvmsyhf1983@hotmail.com'),
(68521, 68506, NULL, NULL, NULL, NULL, 'Motrin | Order Online', NULL, 'ubaqopg1986@hotmail.com'),
(68522, 68507, NULL, NULL, NULL, NULL, 'Biaxin | Buy Xl Online', NULL, 'jdloxomm1984@gmail.com'),
(68523, 68508, NULL, NULL, NULL, NULL, 'Requip | Purchase Restless Leg', NULL, 'wlgiwrf1977@hotmail.com'),
(68524, 68509, NULL, NULL, NULL, NULL, 'Midamor | Purchase Word', NULL, 'ohdejgsw1975@gmail.com'),
(68525, 68510, NULL, NULL, NULL, NULL, 'Cyklokapron | Buy Uk', NULL, 'ocyvaum1984@gmail.com'),
(68526, 68511, NULL, NULL, NULL, NULL, 'Toradol |  Back Order', NULL, 'jwigipwa1976@yahoo.com'),
(68527, 68512, NULL, NULL, NULL, NULL, 'Lamisil | Buy', NULL, 'blpkilcwf1976@gmail.com'),
(68528, 68513, NULL, NULL, NULL, NULL, 'Tadacip | Buy Canada', NULL, 'utbgoue1985@hotmail.com'),
(68529, 68514, NULL, NULL, NULL, NULL, 'Clomipramine | Where To Buy', NULL, 'pvnvbel1977@hotmail.com'),
(68530, 68515, NULL, NULL, NULL, NULL, 'Zebeta | Order Beta', NULL, 'zpgvormy1977@hotmail.com'),
(68531, 68516, NULL, NULL, NULL, NULL, 'Promethazine |  Back Order', NULL, 'ffbwgtxh1985@hotmail.com'),
(68532, 68517, NULL, NULL, NULL, NULL, 'Prozac | Buy Pet', NULL, 'jloodyprb1975@gmail.com'),
(68533, 68518, NULL, NULL, NULL, NULL, 'Levitra | Buy South Africa', NULL, 'gbqpnletg1988@yahoo.com'),
(68534, 68519, NULL, NULL, NULL, NULL, 'Lasix | Buy Online Australia', NULL, 'qkqnzafx1979@gmail.com'),
(68535, 68520, NULL, NULL, NULL, NULL, 'Glucophage |  To Buy', NULL, 'csxebjkr1978@hotmail.com'),
(68536, 68521, NULL, NULL, NULL, NULL, 'Pulmicort | Where To Buy', NULL, 'vlkvjag1978@gmail.com'),
(68537, 68522, NULL, NULL, NULL, NULL, 'Lamictal | Where Can I Buy', NULL, 'vuxpteh1985@gmail.com'),
(68538, 68523, NULL, NULL, NULL, NULL, 'Yasmin | Buy Pill Online Uk', NULL, 'nmqzegk1975@yahoo.com'),
(68539, 68524, NULL, NULL, NULL, NULL, 'Cephalexin | Buy Keflex 500 Mg', NULL, 'tjejngeje1987@hotmail.com'),
(68540, 68525, NULL, NULL, NULL, NULL, 'Ventolin | Buy Inhaler Singapore', NULL, 'fcljnejou1987@hotmail.com'),
(68541, 68526, NULL, NULL, NULL, NULL, 'Naltrexone | Buy 30Mg In Ireland', NULL, 'zqfpskhic1983@yahoo.com'),
(68542, 68527, NULL, NULL, NULL, NULL, 'Metformin | Order Medication', NULL, 'rmmxssjpr1987@gmail.com'),
(68543, 68528, NULL, NULL, NULL, NULL, 'Celexa | Buy Online Cheap', NULL, 'fdpvaavna1988@yahoo.com'),
(68544, 68529, NULL, NULL, NULL, NULL, 'Stromectol | Purchase For Lice', NULL, 'bbbahdhs1982@yahoo.com'),
(68545, 68530, NULL, NULL, NULL, NULL, 'Zocor | Order', NULL, 'tsxxcdyq1983@hotmail.com'),
(68547, 68532, NULL, NULL, NULL, NULL, 'Zyloprim | Buy Allopurinol 100Mg Uk', NULL, 'uachlvtk1987@gmail.com'),
(68548, 68533, NULL, NULL, NULL, NULL, 'Cardura | Purchase', NULL, 'cylanct1980@yahoo.com'),
(68549, 68534, NULL, NULL, NULL, NULL, 'Ditropan | Order Online', NULL, 'kyditelr1987@hotmail.com'),
(68550, 68535, NULL, NULL, NULL, NULL, 'Nolvadex | Buy Novedex Xt', NULL, 'elixxatv1977@yahoo.com'),
(68551, 68536, NULL, NULL, NULL, NULL, 'Medrol | Purchase Dose Pack', NULL, 'eafdxrxyy1980@hotmail.com'),
(68552, 68537, NULL, NULL, NULL, NULL, 'Medrol | Buy Neo', NULL, 'otccbek1983@yahoo.com'),
(68553, 68538, NULL, NULL, NULL, NULL, 'Suprax | Order', NULL, 'slbeupzd1976@yahoo.com'),
(68554, 68539, NULL, NULL, NULL, NULL, 'Floxin | Order', NULL, 'sdyfqav1979@yahoo.com'),
(68555, 68540, NULL, NULL, NULL, NULL, 'Pyridium | Order Side', NULL, 'frqawcez1981@hotmail.com'),
(68556, 68541, NULL, NULL, NULL, NULL, 'Proventil | Order Hfa Online', NULL, 'oeruwmvzt1985@yahoo.com'),
(68557, 68542, NULL, NULL, NULL, NULL, 'Tofranil | Purchase For Ibs', NULL, 'pyqhbkddv1987@hotmail.com'),
(68558, 68543, NULL, NULL, NULL, NULL, 'Imitrex | Buy 100Mg', NULL, 'egvniehnj1975@yahoo.com'),
(68559, 68544, NULL, NULL, NULL, NULL, 'Altace | Purchase 10', NULL, 'otvngpozk1985@hotmail.com'),
(68560, 68545, NULL, NULL, NULL, NULL, 'Depakote | Purchase', NULL, 'hcqggcq1984@gmail.com'),
(68561, 68546, NULL, NULL, NULL, NULL, 'Combivent | Buy From India', NULL, 'ahvaljh1985@yahoo.com'),
(68562, 68547, NULL, NULL, NULL, NULL, 'Atarax | Can You Buy Otc', NULL, 'rvjvgiov1984@gmail.com'),
(68563, 68548, NULL, NULL, NULL, NULL, 'Zyban | Buy In Uk', NULL, 'wuyasjws1987@hotmail.com'),
(68564, 68549, NULL, NULL, NULL, NULL, 'Robaxin | Buy From Canada', NULL, 'pgiglkiql1988@gmail.com'),
(68565, 68550, NULL, NULL, NULL, NULL, 'Metoclopramide |  To Buy', NULL, 'uiwkfkck1975@hotmail.com'),
(68566, 68551, NULL, NULL, NULL, NULL, 'Benicar | Buy 40 Mg', NULL, 'etvimwzaa1981@hotmail.com'),
(68567, 68552, NULL, NULL, NULL, NULL, 'Enalapril |  Maleate Purchase', NULL, 'ralxvrrdh1982@gmail.com'),
(68568, 68553, NULL, NULL, NULL, NULL, 'Enalapril | Buy', NULL, 'bekgbwsur1986@hotmail.com'),
(68569, 68554, NULL, NULL, NULL, NULL, 'Vasotec | Buy Enalapril 5Mg', NULL, 'rowxiqfr1978@gmail.com'),
(68570, 68555, NULL, NULL, NULL, NULL, 'Vasotec | Purchase Enalapril', NULL, 'vexsnwbo1978@gmail.com'),
(68571, 68556, NULL, NULL, NULL, NULL, 'Maxalt | Buy Generic', NULL, 'ubtovmnu1977@gmail.com'),
(68572, 68557, NULL, NULL, NULL, NULL, 'Bactroban | Buy Topical', NULL, 'bsszzxih1978@gmail.com'),
(68573, 68558, NULL, NULL, NULL, NULL, 'Diamox | Buy In Lima', NULL, 'jmkjhfbta1977@hotmail.com'),
(68574, 68559, NULL, NULL, NULL, NULL, 'Tinidazole | Where To Buy 500Mg', NULL, 'sjqatnqgs1976@hotmail.com'),
(68575, 68560, NULL, NULL, NULL, NULL, 'Hyzaar | Order 100', NULL, 'xdhuhkyy1988@hotmail.com'),
(68576, 68561, NULL, NULL, NULL, NULL, 'Provera | Can You Order Online', NULL, 'ujhvaepov1982@hotmail.com'),
(68577, 68562, NULL, NULL, NULL, NULL, 'Biaxin | Purchase Antibiotic', NULL, 'fczyrjraz1988@gmail.com'),
(68578, 68563, NULL, NULL, NULL, NULL, 'Lamictal | Purchase For Depression', NULL, 'ixjsjkfk1982@yahoo.com'),
(68579, 68564, NULL, NULL, NULL, NULL, 'Naltrexone | Buy In Spain', NULL, 'vfsqrgxk1987@gmail.com'),
(68580, 68565, NULL, NULL, NULL, NULL, 'Naltrexone | Buy In The Uk', NULL, 'llbnwts1981@hotmail.com'),
(68581, 68566, NULL, NULL, NULL, NULL, 'ประกันภัยรถยนต์', NULL, 'EmmaBurkhartn760@hotmail.com'),
(68582, 68567, NULL, NULL, NULL, NULL, 'student', 'Universiti Kebangsaan Malaysia', 'izzahabdghani@gmail.com'),
(68583, 68568, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Kebangsaan Malaysia', 'shiplu56@gmail.com'),
(68584, 68569, NULL, NULL, NULL, NULL, 'ENT/ Otorhinolaryngology', 'KPJ Ampang Puteri  Specialist Hospital', 'dr_aminuddin@kpjampang.com '),
(68585, 68570, NULL, NULL, NULL, NULL, 'Lecturer', 'Universiti Kebangsaan Malaysia', 'ruszyidrus@gmail.com'),
(68587, 68572, NULL, NULL, NULL, NULL, 'Naltrexone | Buy Low Dose India', NULL, 'rjlgcpf1982@hotmail.com'),
(68588, 68573, NULL, NULL, NULL, NULL, 'ประกันบิ๊กไบค์', NULL, 'EmmaBurkhartn760@hotmail.com');

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(764, 761, 'MS0001', NULL, NULL, 775, 1, NULL, 774, 1, NULL, 776, 1, NULL, 777, 1, NULL, 'Tai Chi in pre-menopausal women', NULL, NULL),
(766, 761, 'MS0001', NULL, NULL, 775, 1, NULL, 774, 1, NULL, 776, 1, NULL, 777, 1, NULL, 'Tai Chi in pre-menopausal women', 'yes ', 'yes '),
(905, 900, 'MS0002', NULL, NULL, 998, 1, NULL, 994, 1, NULL, 995, 1, NULL, 997, 1, NULL, 'Automated Thyroid Autoantibody Assay', 'no', 'yes '),
(908, 903, 'MS0003', NULL, NULL, 1003, 1, NULL, 1001, 1, NULL, 1002, 1, NULL, 1007, 1, NULL, '3p25 aneusomy in follicular thyroid neoplasms', 'no', 'yes '),
(912, 907, 'MS0006', NULL, NULL, 1026, 1, NULL, 1023, 1, NULL, 1024, 1, NULL, 1025, 1, NULL, 'Testing Submission', 'yes ', 'yes '),
(951, 946, 'MS0007', NULL, NULL, 1070, 1, NULL, 1066, 1, NULL, 1067, 1, NULL, 1069, 1, NULL, 'test', 'no', 'yes '),
(954, 949, 'MS0008', NULL, NULL, 1079, 1, NULL, 1076, 1, NULL, 1077, 1, NULL, 1078, 1, NULL, '-', 'no', 'yes '),
(956, 951, 'MS0009', NULL, NULL, 1086, 1, NULL, 1083, 1, NULL, 1084, 1, NULL, 1085, 1, NULL, 'Biliary Ascariasis from a rural setting: A case study', 'no', 'yes '),
(961, 956, 'MS0010', NULL, NULL, 1103, 1, NULL, 1100, 1, NULL, 1101, 1, NULL, 1102, 1, NULL, 'Knowledge impact in dengue management', 'no', 'yes '),
(964, 959, 'MS0011', NULL, NULL, 1107, 1, NULL, 1104, 1, NULL, 1105, 1, NULL, 1106, 1, NULL, 'photoprotective measures of sunscreens', 'no', 'yes '),
(966, 961, 'MS0012', NULL, NULL, 1116, 1, NULL, 1113, 1, NULL, 1114, 1, NULL, 1115, 1, NULL, 'Management of surge in critically ill patients in a tertiary teaching hospital', 'no', 'yes '),
(973, 968, 'MS0013', NULL, NULL, 1134, 1, NULL, 1131, 1, NULL, 1132, 1, NULL, 1133, 1, NULL, 'stab wound causing delayed left ventricular thrombus embolization', 'no', 'yes '),
(976, 971, 'MS0014', NULL, NULL, 1139, 1, NULL, 1137, 1, NULL, 1136, 1, NULL, 1138, 1, NULL, 'Haemodialysis Affects Corneal Thickness but Not Intraocular Pressure Measurements', 'no', 'yes '),
(977, 972, 'MS0015', NULL, NULL, 1153, 1, NULL, 1150, 1, NULL, 1151, 1, NULL, 1152, 1, NULL, 'injuries during national silat championship', 'no', 'yes '),
(979, 974, 'MS0016', NULL, NULL, 1157, 1, NULL, 1155, 1, NULL, 1156, 1, NULL, 1158, 1, NULL, 'The Determinants of Practicing a Healthy Lifestyle', 'yes ', 'yes '),
(980, 975, 'MS0017', NULL, NULL, 1160, 1, NULL, 1159, 1, NULL, 1161, 1, NULL, 1162, 1, NULL, 'HONEY: FOOD OR MEDICINE?', 'no', 'yes '),
(981, 976, 'MS0018', NULL, NULL, 1165, 1, NULL, 1164, 1, NULL, 1166, 1, NULL, 1167, 1, NULL, 'Bilateral retinal occlusive vasculitis: infective or immunogenic TB?', 'no', 'yes '),
(982, 977, 'MS0019', NULL, NULL, 1173, 1, NULL, 1169, 1, NULL, 1171, 1, NULL, 1172, 1, NULL, 'Masquerade syndrome: an ocular involvement of lung cancer', 'no', 'yes '),
(984, 979, 'MS0020', NULL, NULL, 1192, 1, NULL, 1189, 1, NULL, 1190, 1, NULL, 1191, 1, NULL, 'test', 'no', 'yes '),
(992, 968, 'MS0013', NULL, NULL, 1225, 1, NULL, 1222, 1, NULL, 1223, 1, NULL, 1224, 1, NULL, 'stab wound causing delayed left ventricular thrombus embolization', 'no', 'yes '),
(993, 987, 'MS0021', NULL, NULL, 1230, 1, NULL, 1227, 1, NULL, 1228, 1, NULL, 1229, 1, NULL, 'Functional Recovery Of Stroke Patients At 3 Months Post Admission', 'no', 'yes '),
(1027, 961, 'MS0012', NULL, NULL, 1276, 1, NULL, 1273, 1, NULL, 1274, 1, NULL, 1275, 1, NULL, 'EDOC in a tertiary teaching hospital', 'no', 'yes '),
(1029, 1022, 'MS0022', NULL, NULL, 1284, 1, NULL, 1282, 1, NULL, 1281, 1, NULL, 1283, 1, NULL, 'Obesity and depression', 'no', 'yes '),
(1036, 1029, 'MS0023', NULL, NULL, 1294, 1, NULL, 1291, 1, NULL, 1292, 1, NULL, 1293, 1, NULL, 'CKD stage 3b in diabetics', 'no', 'yes '),
(1074, 1067, 'MS0024', NULL, NULL, 1309, 1, NULL, 1311, 1, NULL, 1307, 1, NULL, 1308, 1, NULL, 'Thalassaemia Screening Among Healthy Blood Donors', 'no', 'yes '),
(1077, 1070, 'MS0025', NULL, NULL, 1316, 1, NULL, 1313, 1, NULL, 1314, 1, NULL, 1315, 1, NULL, 'Stroke awareness among the public', 'no', 'yes '),
(1084, 1077, 'MS0026', NULL, NULL, 1331, 1, NULL, 1329, 1, NULL, 1328, 1, NULL, 1330, 1, NULL, 'QUS for bone assessment', 'no', 'yes '),
(1087, 1080, 'MS0027', NULL, NULL, 1346, 1, NULL, 1333, 1, NULL, 1334, 1, NULL, 1335, 1, NULL, 'Automated external defibrillator (AED) use among paramedics', 'no', 'yes '),
(1088, 1067, 'MS0024', NULL, NULL, 1344, 1, NULL, 1311, 1, NULL, 1307, 1, NULL, 1343, 1, NULL, 'Thalassaemia Screening Among Healthy Blood Donors', 'no', 'yes '),
(1095, 1087, 'MS0028', NULL, NULL, 1358, 1, NULL, 1354, 1, NULL, 1355, 1, NULL, 1357, 1, NULL, 'Total contact plaster boot for ulcers', 'yes ', 'yes '),
(1139, 1131, 'MS0029', NULL, NULL, 1392, 1, NULL, 1391, 1, NULL, 1390, 1, NULL, 1389, 1, NULL, 'Postnatal Urinary Incontinence', 'no', 'yes '),
(1145, 1137, 'MS0030', NULL, NULL, 1622, 1, NULL, 1619, 1, NULL, 1620, 1, NULL, 1621, 1, NULL, 'midazolam use in emergency department', 'no', 'yes '),
(1152, 1144, 'MS0031', NULL, NULL, 1492, 1, NULL, 1403, 1, NULL, 1457, 1, NULL, 1464, 1, NULL, 'Static lactate and lactate clearance with 28 day sepsis mortality', 'no', 'yes '),
(1154, 1146, 'MS0032', NULL, NULL, 1422, 1, NULL, 1419, 1, NULL, 1420, 1, NULL, 1421, 1, NULL, 'Cardiovascular fitness among COPD patients', 'yes ', 'yes '),
(1157, 1149, 'MS0033', NULL, NULL, 1428, 1, NULL, 1431, 1, NULL, 1430, 1, NULL, 1434, 1, NULL, 'Managing ‘Schizophrenia In the Midst of Schizophrenia’: The Role of Assertive Community Treatment ', 'no', 'yes '),
(1160, 1152, 'MS0034', NULL, NULL, 1439, 1, NULL, 1437, 1, NULL, 1436, 1, NULL, 1438, 1, NULL, 'Orogastric lavage for poisoning', 'yes ', 'yes '),
(1162, 1154, 'MS0035', NULL, NULL, 1490, 1, NULL, 1487, 1, NULL, 1488, 1, NULL, 1489, 1, NULL, 'PRIMARY LOW GRADE PAPILLARY ADENOCARCINOMA OF NASOPHARYNX', 'no', 'yes '),
(1221, 1213, 'MS0036', NULL, NULL, 1498, 1, NULL, 1495, 1, NULL, 1496, 1, NULL, 1497, 1, NULL, 'Patient Education Package for Pain', 'no', 'yes '),
(1269, 1261, 'MS0037', NULL, NULL, 1519, 1, NULL, 1521, 1, NULL, 1517, 1, NULL, 1518, 1, NULL, 'Wobble Board Training in Unstable Ankle Joint', 'yes ', 'yes '),
(1279, 1271, 'MS0038', NULL, NULL, 1528, 1, NULL, 1530, 1, NULL, 1526, 1, NULL, 1527, 1, NULL, 'Radiation Retinopathy in Nasopharyngeal Carcinoma', 'no', 'yes '),
(1290, 1282, 'MS0039', NULL, NULL, 1536, 1, NULL, 1531, 1, NULL, 1532, 1, NULL, 1533, 1, NULL, 'Nutrient foramina of metatarsals', 'yes ', 'yes '),
(1293, 1285, 'MS0040', NULL, NULL, 1553, 1, NULL, 1550, 1, NULL, 1551, 1, NULL, 1552, 1, NULL, '"Focal" Cardiac Concussion', 'no', 'yes '),
(1299, 1291, 'MS0041', NULL, NULL, 1572, 1, NULL, 1569, 1, NULL, 1570, 1, NULL, 1571, 1, NULL, 'Depression among adolescent', 'no', 'yes '),
(1301, 1293, 'MS0042', NULL, NULL, 1577, 1, NULL, 1580, 1, NULL, 1575, 1, NULL, 1576, 1, NULL, 'Homicide-suicide: A Case Report', 'no', 'yes '),
(1308, 1300, 'MS0043', NULL, NULL, 1590, 1, NULL, 1586, 1, NULL, 1588, 1, NULL, 1589, 1, NULL, 'IDENTIFICATION OF MAJOR AND MINOR ALLERGENS OF MUD CRAB (SCYLLA SERRATA)', 'no', 'yes '),
(1309, 1293, 'MS0042', NULL, NULL, 1600, 1, NULL, 1597, 1, NULL, 1598, 1, NULL, 1599, 1, NULL, 'Homicide-suicide: A Case Report', 'no', 'yes '),
(1364, 1355, 'MS0044', NULL, NULL, 1626, 1, NULL, 1623, 1, NULL, 1624, 1, NULL, 1625, 1, NULL, 'This is a case report illustrating a missed fungaemia which lead to a rare fungal eye infection that potentially be a vision threatening, nonetheless, with prompt intervention, such dreadful complication could still be salvageable.', 'no', 'yes '),
(1716, 1707, 'MS0045', NULL, NULL, 1641, 1, NULL, 1638, 1, NULL, 1639, 1, NULL, 1640, 1, NULL, 'Ultrasound as a triage tool to rule out fracture', 'no', 'yes '),
(6035, 6026, 'MS0048', NULL, NULL, 1819, 1, NULL, 1817, 1, NULL, 1818, 1, NULL, 1815, 1, NULL, 'acute ashtma re visit to emergency department', 'no', 'yes '),
(6042, 6033, 'MS0049', NULL, NULL, 1686, 1, NULL, 1683, 1, NULL, 1684, 1, NULL, 1685, 1, NULL, 'PfNT3, Plasmodium falciparum Nucleoside permease', 'yes ', 'yes '),
(6092, 6083, 'MS0050', NULL, NULL, 1760, 1, NULL, 1757, 1, NULL, 1758, 1, NULL, 1759, 1, NULL, 'Test of Sex Estimation Equations on Carpal Bones in Thais', 'yes ', 'yes '),
(6093, 6084, 'MS0051', NULL, NULL, 1766, 1, NULL, 1763, 1, NULL, 1764, 1, NULL, 1765, 1, NULL, 'The Accuracy of Sex Estimation on Metatarsal Bones in Thais', 'yes ', 'yes '),
(6106, 6097, 'MS0052', NULL, NULL, 1774, 1, NULL, 1771, 1, NULL, 1772, 1, NULL, 1773, 1, NULL, 'DAMA', 'yes ', 'yes '),
(6111, 6102, 'MS0053', NULL, NULL, 1784, 1, NULL, 1783, 1, NULL, 1782, 1, NULL, 1781, 1, NULL, 'OHCA', 'yes ', 'yes '),
(6309, 6300, 'MS0054', NULL, NULL, 1791, 1, NULL, 1792, 1, NULL, 1790, 1, NULL, 1788, 1, NULL, 'Yogasana: health benefits', 'yes ', 'yes '),
(6556, 6547, 'MS0055', NULL, NULL, 1796, 1, NULL, 1793, 1, NULL, 1794, 1, NULL, 1795, 1, NULL, 'Video assisted learning ', 'no', 'yes '),
(6648, 6639, 'MS0056', NULL, NULL, 1807, 1, NULL, 1803, 1, NULL, 1804, 1, NULL, 1805, 1, NULL, 'MALAYSIAN NASAL POLYPS', 'no', 'yes '),
(7009, 7000, 'MS0057', NULL, NULL, 1813, 1, NULL, 1810, 1, NULL, 1811, 1, NULL, 1812, 1, NULL, 'Subglottic cyst', 'no', 'yes '),
(7032, 7023, 'MS0058', NULL, NULL, 1822, 1, NULL, 1821, 1, NULL, 1824, 1, NULL, 1825, 1, NULL, 'Patient-Controlled Analgesia (PCA) for orthopaedic patients', 'no', 'yes '),
(7245, 7236, 'MS0059', NULL, NULL, 1849, 1, NULL, 1868, 1, NULL, 1869, 1, NULL, 1844, 1, NULL, 'Linguistic validation of HypoSRQ', 'no', 'yes '),
(7248, 7239, 'MS0060', NULL, NULL, 1861, 1, NULL, 1858, 1, NULL, 1859, 1, NULL, 1860, 1, NULL, 'MALAYSIAN NASAL POLYPS', 'no', 'yes '),
(7253, 7244, 'MS0061', NULL, NULL, 1881, 1, NULL, 1873, 1, NULL, 1874, 1, NULL, 1875, 1, NULL, 'Quantification of Bone Histomrphometric Parameters', 'yes ', 'yes '),
(7293, 7284, 'MS0062', NULL, NULL, 2625, 1, NULL, 2623, 1, NULL, 2624, 1, NULL, 1897, 1, NULL, 'BULLAE MISDIAGNOSED AS A PNEUMOTHORAX', 'no', 'yes '),
(7318, 7309, 'MS0063', NULL, NULL, 1982, 1, NULL, 2318, 1, NULL, 1984, 1, NULL, 1983, 1, NULL, 'TEMPORAL BONE GCT', 'yes ', 'yes '),
(7319, 7310, 'MS0064', NULL, NULL, 1969, 1, NULL, 1960, 1, NULL, 1967, 1, NULL, 1968, 1, NULL, 'Colour vision status of staff working with hospital radiation', 'no', 'yes '),
(7328, 7319, 'MS0065', NULL, NULL, 2032, 1, NULL, 2029, 1, NULL, 2030, 1, NULL, 2031, 1, NULL, 'LDH in falciparum malaria', 'yes ', 'yes '),
(7339, 7330, 'MS0066', NULL, NULL, 1986, 1, NULL, 1988, 1, NULL, 1987, 1, NULL, 1989, 1, NULL, 'Effect of 2% cholesterol on blood pressure in ovariectomised rats', 'yes ', 'yes '),
(7363, 7354, 'MS0067', NULL, NULL, 2040, 1, NULL, 2007, 1, NULL, 2039, 1, NULL, 2036, 1, NULL, ' Expression of p53 protein in women within the premenopausal age', 'no', 'yes '),
(13150, 13141, 'MS0068', NULL, NULL, 2015, 1, NULL, 2012, 1, NULL, 2013, 1, NULL, 2014, 1, NULL, 'Ankle injuries', 'no', 'yes '),
(13164, 13155, 'MS0069', NULL, NULL, 2679, 1, NULL, 2017, 1, NULL, 2191, 1, NULL, 2678, 1, NULL, 'Pascal versus conventional retinal photocoagulation', 'no', 'yes '),
(13193, 13184, 'MS0070', NULL, NULL, 2053, 1, NULL, 2047, 1, NULL, 2048, 1, NULL, 2049, 1, NULL, 'MMP-3 in Rheumatoid arthritis', 'no', 'yes '),
(25510, 25501, 'MS0074', NULL, NULL, 2125, 1, NULL, 2122, 1, NULL, 2123, 1, NULL, 2124, 1, NULL, 'Assessment of vascular fitness among high risk young male', 'no', 'yes '),
(25518, 7244, 'MS0061', NULL, NULL, 2815, 1, NULL, 1873, 1, NULL, 2814, 1, NULL, 2816, 1, NULL, 'Quantification of Bone Histomorphometric Parameters', 'yes ', 'yes '),
(25691, 25681, 'MS0075', NULL, NULL, 2154, 1, NULL, 2151, 1, NULL, 2152, 1, NULL, 2153, 1, NULL, 'Ginseng as antihyperglycemic remedy', 'yes ', 'yes '),
(27858, 27848, 'MS0076', NULL, NULL, 2209, 1, NULL, 2201, 1, NULL, 2207, 1, NULL, 2208, 1, NULL, 'Effects of medicinal plant extracts on inflammation and GnRH release  ', 'yes ', 'yes '),
(27876, 27866, 'MS0077', NULL, NULL, 2299, 1, NULL, 2296, 1, NULL, 2297, 1, NULL, 2298, 1, NULL, 'Solar Retinopathy. A Case Report', 'no', 'yes '),
(27880, 27870, 'MS0078', NULL, NULL, 2364, 1, NULL, 2363, 1, NULL, 2365, 1, NULL, 2305, 1, NULL, 'Neuroprotective effect of rice and corn extracts against neurotoxicity', 'yes ', 'yes '),
(27934, 27924, 'MS0079', NULL, NULL, 2316, 1, NULL, 2314, 1, NULL, 2315, 1, NULL, 2317, 1, NULL, 'Tocotrienol on lead-induced brain toxicity', 'yes ', 'yes '),
(28928, 28918, 'MS0080', NULL, NULL, 2324, 1, NULL, 2322, 1, NULL, 2321, 1, NULL, 2323, 1, NULL, 'Pattern of PCV: National University of Malaysia (UKMMC) Experience', 'no', 'yes '),
(29419, 29409, 'MS0081', NULL, NULL, 2353, 1, NULL, 2350, 1, NULL, 2351, 1, NULL, 2352, 1, NULL, 'Gelam Honey and its therapeutic properties', 'yes ', 'yes '),
(29420, 29410, 'MS0082', NULL, NULL, 2362, 1, NULL, 2354, 1, NULL, 2355, 1, NULL, 2361, 1, NULL, 'Management of fifth metatarsal fracture', 'no', 'yes '),
(29551, 29541, 'MS0083', NULL, NULL, 2370, 1, NULL, 2368, 1, NULL, 2369, 1, NULL, 2367, 1, NULL, 'Penile amputation, auditory hallucination in schiophrenia, Malaysia', 'no', 'yes '),
(29584, 29574, 'MS0084', NULL, NULL, 2374, 1, NULL, 2371, 1, NULL, 2372, 1, NULL, 2373, 1, NULL, 'CAM usage among diabetics', 'no', 'yes '),
(29692, 29682, 'MS0085', NULL, NULL, 2389, 1, NULL, 2387, 1, NULL, 2388, 1, NULL, 2390, 1, NULL, 'High Intraocular Pressure and Obstructive Sleep Apnea (OSA) ', 'no', 'yes '),
(29697, 29687, 'MS0086', NULL, NULL, 2399, 1, NULL, 2396, 1, NULL, 2397, 1, NULL, 2398, 1, NULL, 'Tracheostomy mismanagement causing unusual complication', 'no', 'yes '),
(29702, 29692, 'MS0087', NULL, NULL, 2423, 1, NULL, 2414, 1, NULL, 2415, 1, NULL, 2422, 1, NULL, 'ORBITAL CELLULITIS FROM UNTREATED CONJUNCTIVAL WOUND', 'no', 'yes '),
(29760, 29750, 'MS0088', NULL, NULL, 2431, 1, NULL, 2435, 1, NULL, 2436, 1, NULL, 2430, 1, NULL, 'CNPAS as a Rare Manifestation of Trisomy 8 Mosaicsm', 'no', 'yes '),
(29804, 29794, 'MS0089', NULL, NULL, 2470, 1, NULL, 2439, 1, NULL, 2440, 1, NULL, 2441, 1, NULL, 'Effects of Eurycoma longifolia on fracture healing', 'no', 'yes '),
(29805, 29795, 'MS0090', NULL, NULL, 2459, 1, NULL, 2458, 1, NULL, 2464, 1, NULL, 2465, 1, NULL, 'Shake till you break', 'no', 'yes '),
(29864, 29854, 'MS0091', NULL, NULL, 2476, 1, NULL, 2477, 1, NULL, 2479, 1, NULL, 2478, 1, NULL, 'SENSITIVITY AND SPECIFICITY OF PPGF IN CVD RISK STRATIFICATION ', 'no', 'yes '),
(29964, 29954, 'MS0092', NULL, NULL, 2484, 1, NULL, 2480, 1, NULL, 2481, 1, NULL, 2482, 1, NULL, 'CMV retinitis in dermatomyositis ', 'no', 'yes '),
(29969, 29959, 'MS0093', NULL, NULL, 2493, 1, NULL, 2489, 1, NULL, 2490, 1, NULL, 2491, 1, NULL, 'CNPAS as a Rare Manifestation of Trisomy 8 Mosaicsm', 'no', 'yes '),
(31577, 31567, 'MS0094', NULL, NULL, 2503, 1, NULL, 2504, 1, NULL, 2499, 1, NULL, 2500, 1, NULL, 'Vitamin E Promotes Wound Healing', 'no', 'yes '),
(32699, 32689, 'MS0095', NULL, NULL, 2512, 1, NULL, 2508, 1, NULL, 2509, 1, NULL, 2510, 1, NULL, 'ADULT ATTENTION DEFICIT DISORDER', 'no', 'yes '),
(35944, 35934, 'MS0096', NULL, NULL, 2516, 1, NULL, 2513, 1, NULL, 2514, 1, NULL, 2515, 1, NULL, 'BICLONAL GAMMOPATHY: A RARE CASE', 'no', 'yes '),
(38204, 38194, 'MS0097', NULL, NULL, 2536, 1, NULL, 2550, 1, NULL, 2533, 1, NULL, 2534, 1, NULL, 'QOL of neovascular-AMD patients after intravitreal Ranibizumab.', 'no', 'yes '),
(38219, 38209, 'MS0098', NULL, NULL, 2677, 1, NULL, 2546, 1, NULL, 2675, 1, NULL, 2676, 1, NULL, 'Antibacterial properties of Euphorbia tirucalli stem extracts', 'no', 'yes '),
(41571, 41561, 'MS0099', NULL, NULL, 2563, 1, NULL, 2562, 1, NULL, 2557, 1, NULL, 2558, 1, NULL, 'Genetic diversity of H. pylori', 'no', 'yes '),
(42049, 42039, 'MS0100', NULL, NULL, 2565, 1, NULL, 2570, 1, NULL, 2567, 1, NULL, 2566, 1, NULL, 'BCC from epidermal naevus', 'no', 'yes '),
(42879, 42869, 'MS0101', NULL, NULL, 2574, 1, NULL, 2571, 1, NULL, 2572, 1, NULL, 2573, 1, NULL, 'PARENTING STRESS IN ASD', 'no', 'yes '),
(46094, 46084, 'MS0102', NULL, NULL, 2585, 1, NULL, 2583, 1, NULL, 2584, 1, NULL, 2586, 1, NULL, 'The study of Cumin oil essence on the function of FUM1 genes in Fusarium verticillioides', 'no', 'yes '),
(46452, 46442, 'MS0103', NULL, NULL, 2590, 1, NULL, 2587, 1, NULL, 2588, 1, NULL, 2589, 1, NULL, 'comparing sterofundin to normal saline in managing DKA', 'no', 'yes '),
(49280, 49270, 'MS0104', NULL, NULL, 2596, 1, NULL, 2599, 1, NULL, 2600, 1, NULL, 2597, 1, NULL, 'Scrotal basal cell carcinoma ', 'no', 'yes '),
(50713, 50703, 'MS0105', NULL, NULL, 2611, 1, NULL, 2608, 1, NULL, 2609, 1, NULL, 2610, 1, NULL, 'Subhypnotic doses of propofol to prevent intrathecal morphine-induced pruritus', 'no', 'yes '),
(50782, 27866, 'MS0077', NULL, NULL, 2613, 1, NULL, 2296, 1, NULL, 2297, 1, NULL, 2612, 1, NULL, 'Solar Retinopathy. A Case Report', 'no', 'yes '),
(51616, 29687, 'MS0086', NULL, NULL, 2632, 1, NULL, 2631, 1, NULL, 2630, 1, NULL, 2629, 1, NULL, 'Tracheostomy mismanagement causing unusual complication', 'no', 'yes '),
(51992, 51980, 'MS0106', NULL, NULL, 2641, 1, NULL, 2633, 1, NULL, 2634, 1, NULL, 2635, 1, NULL, 'Urethral Masson’s tumourlim', 'no', 'yes '),
(52835, 52823, 'MS0107', NULL, NULL, 2712, 1, NULL, 2649, 1, NULL, 2642, 1, NULL, 2706, 1, NULL, 'Running title: Medical embalming in tropical climate', 'no', 'yes '),
(53307, 53295, 'MS0108', NULL, NULL, 2653, 1, NULL, 2650, 1, NULL, 2651, 1, NULL, 2652, 1, NULL, 'Medication wastages and its disposal amongst patients in SSB Hospital', 'yes ', 'yes '),
(53560, 53548, 'MS0109', NULL, NULL, 2659, 1, NULL, 2656, 1, NULL, 2657, 1, NULL, 2658, 1, NULL, 'Hepatocytes 11β-Hydroxysteroid Dehydrogenase Type 1Enzyme in Metabolic Syndrome Rat Model.', 'yes ', 'yes '),
(54252, 54240, 'MS0110', NULL, NULL, 2669, 1, NULL, 2666, 1, NULL, 2668, 1, NULL, 2667, 1, NULL, 'A case report.', 'no', 'yes '),
(54378, 54366, 'MS0111', NULL, NULL, 2673, 1, NULL, 2670, 1, NULL, 2671, 1, NULL, 2672, 1, NULL, '11β-Hydroxysteroid Dehydrogenase Enzyme in Metabolic Syndrome', 'no', 'yes '),
(55059, 53295, 'MS0108', NULL, NULL, 2687, 1, NULL, 2650, 1, NULL, 2651, 1, NULL, 2686, 1, NULL, 'Medication wastages and its disposal amongst patients in SSB Hospital', 'yes ', 'yes '),
(55169, 55156, 'MS0112', NULL, NULL, 2693, 1, NULL, 2690, 1, NULL, 2691, 1, NULL, 2692, 1, NULL, 'Parenting Stress in Thalassaemia', 'no', 'yes '),
(64126, 64113, 'MS0113', NULL, NULL, 3105, 1, NULL, 2740, 1, NULL, 2761, 1, NULL, 2742, 1, NULL, 'Msi2 protein expression in AML and cytogenetics', 'no', 'yes '),
(65400, 65387, 'MS0114', NULL, NULL, 2772, 1, NULL, 2769, 1, NULL, 2770, 1, NULL, 2771, 1, NULL, 'corneal scrape specimen into bactec bottle', 'no', 'yes '),
(66263, 66250, 'MS0115', NULL, NULL, 2779, 1, NULL, 2776, 1, NULL, 2777, 1, NULL, 2778, 1, NULL, 'Nasopharyngeal Tuberculosis', 'no', 'yes '),
(67410, 67397, 'MS0116', NULL, NULL, 2794, 1, NULL, 2784, 1, NULL, 2785, 1, NULL, 2788, 1, NULL, 'Age Estimation from Clavicle by Histomorphometry', 'yes ', 'yes '),
(67412, 67399, 'MS0117', NULL, NULL, 2803, 1, NULL, 2790, 1, NULL, 2791, 1, NULL, 2792, 1, NULL, 'Age Estimation in Human Tissues by Aspartic Acid Racemization', 'yes ', 'yes '),
(67414, 67401, 'MS0118', NULL, NULL, 2798, 1, NULL, 2795, 1, NULL, 2796, 1, NULL, 2797, 1, NULL, 'Age estimation from purified elastin of aorta by aspartic acid racemization', 'yes ', 'yes '),
(67416, 67403, 'MS0119', NULL, NULL, 2802, 1, NULL, 2799, 1, NULL, 2800, 1, NULL, 2801, 1, NULL, 'Biological markers associated Osteoarthritis', 'yes ', 'yes '),
(67484, 67471, 'MS0120', NULL, NULL, 2807, 1, NULL, 2804, 1, NULL, 2805, 1, NULL, 2806, 1, NULL, 'missed thoracic fracture', 'no', 'yes '),
(67534, 29692, 'MS0087', NULL, NULL, 2819, 1, NULL, 2414, 1, NULL, 2415, 1, NULL, 2817, 1, NULL, 'ORBITAL CELLULITIS FROM UNTREATED CONJUNCTIVAL WOUND', 'no', 'yes '),
(67548, 67534, 'MS0121', NULL, NULL, 3052, 1, NULL, 2820, 1, NULL, 2821, 1, NULL, 2822, 1, NULL, 'Uncommon variation of RLN in total thyroidectomy risk for RLN injury', 'no', 'yes '),
(67733, 67719, 'MS0122', NULL, NULL, 2933, 1, NULL, 2930, 1, NULL, 2931, 1, NULL, 2932, 1, NULL, 'Antenatal Iron Deficiency in Urban Malaysia', 'no', 'yes '),
(67750, 67736, 'MS0123', NULL, NULL, 3058, 1, NULL, 2943, 1, NULL, 3055, 1, NULL, 3057, 1, NULL, 'Co-infection with Dengue and Leptospirosis', 'no', 'yes '),
(67824, 67810, 'MS0124', NULL, NULL, 2955, 1, NULL, 2956, 1, NULL, 2953, 1, NULL, 2954, 1, NULL, 'Evaluation of Houttuynia cordata Thunb Extract on Human Acute Leukemic Cells', 'yes ', 'yes '),
(67829, 67815, 'MS0125', NULL, NULL, 2961, 1, NULL, 2957, 1, NULL, 2958, 1, NULL, 2959, 1, NULL, 'OPTIMUM DOSE OF STICHOPUS CHLORONOTUS', 'no', 'yes '),
(67857, 67843, 'MS0126', NULL, NULL, 2970, 1, NULL, 2968, 1, NULL, 2971, 1, NULL, 2969, 1, NULL, 'Septal adenoid cystic carcinoma', 'no', 'yes '),
(67859, 67845, 'MS0127', NULL, NULL, 2977, 1, NULL, 2972, 1, NULL, 2973, 1, NULL, 2974, 1, NULL, 'Unusual variant of sphenoplalatine foramen and artery', 'no', 'yes '),
(67862, 67848, 'MS0128', NULL, NULL, 2982, 1, NULL, 2979, 1, NULL, 2980, 1, NULL, 2981, 1, NULL, 'Branchial fistula: Minimal diagnostics work-up. ', 'no', 'yes '),
(67992, 67978, 'MS0129', NULL, NULL, 3036, 1, NULL, 3033, 1, NULL, 3034, 1, NULL, 3035, 1, NULL, 'Gelam honey and Oxidative stress', 'no', 'yes '),
(67996, 67982, 'MS0130', NULL, NULL, 3043, 1, NULL, 3040, 1, NULL, 3041, 1, NULL, 3042, 1, NULL, 'Hyperbaric Oxygen Therapy and Quality of Life', 'no', 'yes '),
(68044, 68030, 'MS0131', NULL, NULL, 3065, 1, NULL, 3062, 1, NULL, 3063, 1, NULL, 3064, 1, NULL, 'A problem of polypharmacy in elderly', 'no', 'yes '),
(68063, 68049, 'MS0132', NULL, NULL, 3079, 1, NULL, 3076, 1, NULL, 3077, 1, NULL, 3078, 1, NULL, 'Evaluation of different intercalating dyes for HPV16 LAMP', 'no', 'yes '),
(68069, 68055, 'MS0133', NULL, NULL, 3086, 1, NULL, 3089, 1, NULL, 3088, 1, NULL, 3087, 1, NULL, 'Treatment of septic hip dislocation with a preserved femoral head', 'no', 'yes '),
(68143, 68129, 'MS0134', NULL, NULL, 3102, 1, NULL, 3096, 1, NULL, 3101, 1, NULL, 3097, 1, NULL, 'RARE ANGIOSARCOMA OF INFERIOR TURBINATE', 'no', 'yes '),
(68147, 68133, 'MS0135', NULL, NULL, 3110, 1, NULL, 3107, 1, NULL, 3108, 1, NULL, 3109, 1, NULL, 'Msi2 protein expression in AML and cytogenetics', 'no', 'yes '),
(68157, 68143, 'MS0136', NULL, NULL, 3138, 1, NULL, 3135, 1, NULL, 3136, 1, NULL, 3137, 1, NULL, 'Anatomical variations of supraorbital foramen and notch in Malaysian skulls', 'yes ', 'yes '),
(68253, 68239, 'MS0137', NULL, NULL, 3144, 1, NULL, 3145, 1, NULL, 3140, 1, NULL, 3142, 1, NULL, 'ACRAL METASTASIS', 'no', 'yes '),
(68352, 68338, 'MS0138', NULL, NULL, 3153, 1, NULL, 3150, 1, NULL, 3151, 1, NULL, 3152, 1, NULL, 'SECONDARY EXTRAMEDULLARY PLASMACYTOMA OF THE ORBIT', 'no', 'yes '),
(68353, 68339, 'MS0139', NULL, NULL, 3162, 1, NULL, 3167, 1, NULL, 3160, 1, NULL, 3161, 1, NULL, 'ORBITAL CELLULITIS FROM UNTREATED CONJUNCTIVAL WOUND', 'no', 'yes '),
(68374, 68360, 'MS0140', NULL, NULL, 3172, 1, NULL, 3168, 1, NULL, 3169, 1, NULL, 3171, 1, NULL, 'Fatal firearm in Malaysia.', 'no', 'yes '),
(68506, 68492, 'MS0141', NULL, NULL, 3176, 1, NULL, 3173, 1, NULL, 3174, 1, NULL, 3175, 1, NULL, 'CHOROIDAL THICKNESS POST PHACOEMULSIFICATION', 'yes ', 'yes '),
(68512, 65387, 'MS0114', NULL, NULL, 3195, 1, NULL, 2769, 1, NULL, 3193, 1, NULL, 3194, 1, NULL, 'corneal scrape specimen into bactec bottle', 'no', 'yes '),
(68546, 68531, 'MS0142', NULL, NULL, 3200, 1, NULL, 3197, 1, NULL, 3198, 1, NULL, 3199, 1, NULL, 'BICLONAL GAMMOPATHY IN PLASMA CELL MYELOMA', 'no', 'yes '),
(68586, 68571, 'MS0143', NULL, NULL, 3218, 1, NULL, 3215, 1, NULL, 3216, 1, NULL, 3217, 1, NULL, 'EFFECT OF DERMAL FIBROBLASTS CONDITIONED MEDIUM ON RE-EPITHELIALIZATION', 'no', 'yes ');

-- --------------------------------------------------------

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--
-- Table structure for table `content_type_editors_choice`
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--
-- Table structure for table `content_type_event`
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-- --------------------------------------------------------

--
-- Table structure for table `content_type_issue`
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(994, 988, '7', '2', '2012-00-00T00:00:00', 1268, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}'),
(1037, 1030, '8', '1', '2013-00-00T00:00:00', 1296, 1, 'a:2:{s:3:"alt";s:0:"";s:5:"title";s:0:"";}'),
(1096, 1088, '8', '2', '2013-00-00T00:00:00', NULL, NULL, NULL),
(1163, 1155, '9', '1', '2014-00-00T00:00:00', NULL, NULL, NULL),
(1226, 1218, '9', '2', '2014-00-00T00:00:00', NULL, NULL, NULL),
(1310, 1301, '10', '1', '2015-00-00T00:00:00', NULL, NULL, NULL),
(6043, 6034, '10', '2', '2015-00-00T00:00:00', NULL, NULL, NULL),
(7255, 7246, '11', '1', '2016-00-00T00:00:00', NULL, NULL, NULL),
(67597, 67583, '11', '2', '2016-00-00T00:00:00', NULL, NULL, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_type_op_image`
--

CREATE TABLE IF NOT EXISTS `content_type_op_image` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_embedded_image_embed` longtext,
  `field_op_embedded_image_value` varchar(255) DEFAULT NULL,
  `field_op_embedded_image_provider` varchar(255) DEFAULT NULL,
  `field_op_embedded_image_data` longtext,
  `field_op_embedded_image_version` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_image_credit_value` longtext,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_type_op_image`
--

INSERT INTO `content_type_op_image` (`vid`, `nid`, `field_op_embedded_image_embed`, `field_op_embedded_image_value`, `field_op_embedded_image_provider`, `field_op_embedded_image_data`, `field_op_embedded_image_version`, `field_op_image_credit_value`) VALUES
(34, 31, NULL, NULL, NULL, NULL, 0, 'TEST 1'),
(35, 32, NULL, NULL, NULL, NULL, 0, 'TEST 2');

-- --------------------------------------------------------

--
-- Table structure for table `content_type_package`
--

CREATE TABLE IF NOT EXISTS `content_type_package` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_center_intro_value` longtext,
  `field_center_intro_format` int(10) unsigned DEFAULT NULL,
  `field_center_main_image_fid` int(11) DEFAULT NULL,
  `field_center_main_image_list` tinyint(4) DEFAULT NULL,
  `field_center_main_image_data` text,
  `field_center_title_value` longtext,
  `field_left_intro_value` longtext,
  `field_left_intro_format` int(10) unsigned DEFAULT NULL,
  `field_right_intro_value` longtext,
  `field_right_intro_format` int(10) unsigned DEFAULT NULL,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_type_resource`
--

CREATE TABLE IF NOT EXISTS `content_type_resource` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `content_type_slideshow`
--

CREATE TABLE IF NOT EXISTS `content_type_slideshow` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_op_slideshow_thumb_fid` int(11) DEFAULT NULL,
  `field_op_slideshow_thumb_list` tinyint(4) DEFAULT NULL,
  `field_op_slideshow_thumb_data` text,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `content_type_slideshow`
--

INSERT INTO `content_type_slideshow` (`vid`, `nid`, `field_op_slideshow_thumb_fid`, `field_op_slideshow_thumb_list`, `field_op_slideshow_thumb_data`) VALUES
(36, 33, NULL, NULL, NULL);

-- --------------------------------------------------------

--
-- Table structure for table `content_type_video`
--

CREATE TABLE IF NOT EXISTS `content_type_video` (
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `field_flash_file_fid` int(11) DEFAULT NULL,
  `field_flash_file_list` tinyint(4) DEFAULT NULL,
  `field_flash_file_data` text,
  `field_op_video_thumbnail_fid` int(11) DEFAULT NULL,
  `field_op_video_thumbnail_list` tinyint(4) DEFAULT NULL,
  `field_op_video_thumbnail_data` text,
  PRIMARY KEY (`vid`),
  KEY `nid` (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `context`
--

CREATE TABLE IF NOT EXISTS `context` (
  `name` varchar(255) NOT NULL DEFAULT '',
  `description` varchar(255) NOT NULL DEFAULT '',
  `tag` varchar(255) NOT NULL DEFAULT '',
  `conditions` text,
  `reactions` text,
  `condition_mode` int(11) DEFAULT '0',
  PRIMARY KEY (`name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `context`
--

INSERT INTO `context` (`name`, `description`, `tag`, `conditions`, `reactions`, `condition_mode`) VALUES
('content_manuscript', '', '', 'a:1:{s:4:"node";a:2:{s:6:"values";a:2:{s:16:"conference_paper";s:16:"conference_paper";s:17:"conference_review";s:17:"conference_review";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:2:{s:32:"views--exp-author_center-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:26:"-exp-author_center-block_1";s:6:"region";s:4:"left";s:6:"weight";i:0;}s:32:"views--exp-author_center-block_2";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:26:"-exp-author_center-block_2";s:6:"region";s:4:"left";s:6:"weight";i:1;}}}}', 0),
('openpublish-general-sitewide', '', 'OpenPublish General', 'a:1:{s:8:"sitewide";a:1:{s:6:"values";a:1:{i:1;i:1;}}}', 'a:0:{}', 0),
('openpublish-listing_pages-articles', '', 'OpenPublish Listing Page', 'a:1:{s:5:"views";a:1:{s:6:"values";a:1:{s:8:"articles";s:8:"articles";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:2:{s:35:"views-most_viewed_by_taxonomy-block";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:29:"most_viewed_by_taxonomy-block";s:6:"region";s:5:"right";s:6:"weight";i:0;}s:37:"views-most_commented_articles-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:31:"most_commented_articles-block_1";s:6:"region";s:5:"right";s:6:"weight";i:1;}}}}', 0),
('openpublish-listing_pages-events', '', 'OpenPublish Listing Page', 'a:1:{s:5:"views";a:1:{s:6:"values";a:1:{s:6:"events";s:6:"events";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:1:{s:36:"views-most_viewed_by_node_type-block";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:30:"most_viewed_by_node_type-block";s:6:"region";s:5:"right";s:6:"weight";i:0;}}}}', 0),
('openpublish-listing_pages-frontpage', '', 'OpenPublish Listing Page', 'a:1:{s:4:"path";a:1:{s:6:"values";a:1:{s:7:"<front>";s:7:"<front>";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:3:{s:7:"block-8";a:4:{s:6:"module";s:5:"block";s:5:"delta";i:8;s:6:"region";s:4:"left";s:6:"weight";i:0;}s:34:"views--exp-articles_search-block_2";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:28:"-exp-articles_search-block_2";s:6:"region";s:5:"right";s:6:"weight";i:0;}s:23:"views-slideshow-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:17:"slideshow-block_1";s:6:"region";s:5:"right";s:6:"weight";i:1;}}}}', 0),
('openpublish-multi-content_detail', '', 'OpenPublish Detail Page', 'a:1:{s:4:"node";a:2:{s:6:"values";a:4:{s:7:"article";s:7:"article";s:4:"blog";s:4:"blog";s:5:"event";s:5:"event";s:8:"resource";s:8:"resource";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:0:{}', 0),
('openpublish-node_detail-article', '', 'OpenPublish Detail Page', 'a:1:{s:4:"node";a:2:{s:6:"values";a:1:{s:7:"article";s:7:"article";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:3:{s:21:"views-journal-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:15:"journal-block_1";s:6:"region";s:4:"left";s:6:"weight";i:0;}s:34:"views--exp-articles_search-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:28:"-exp-articles_search-block_1";s:6:"region";s:4:"left";s:6:"weight";i:1;}s:20:"views-archivez-block";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:14:"archivez-block";s:6:"region";s:4:"left";s:6:"weight";i:2;}}}}', 0),
('openpublish-node_detail-author', 'Author Page', 'OpenPublish Detail Page', 'a:1:{s:4:"node";a:2:{s:6:"values";a:1:{s:6:"author";s:6:"author";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:0:{}', 0),
('openpublish-node_detail-blog', '', 'OpenPublish Detail Page', 'a:1:{s:4:"node";a:2:{s:6:"values";a:1:{s:4:"blog";s:4:"blog";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:2:{s:18:"views-news-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:12:"news-block_1";s:6:"region";s:5:"right";s:6:"weight";i:0;}s:28:"views-editors_choice-block_2";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:22:"editors_choice-block_2";s:6:"region";s:5:"right";s:6:"weight";i:1;}}}}', 0),
('openpublish-node_detail-event', '', 'OpenPublish Detail Page', 'a:1:{s:4:"node";a:2:{s:6:"values";a:1:{s:5:"event";s:5:"event";}s:7:"options";a:1:{s:9:"node_form";s:1:"1";}}}', 'a:1:{s:5:"block";a:1:{s:6:"blocks";a:2:{s:18:"views-news-block_1";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:12:"news-block_1";s:6:"region";s:5:"right";s:6:"weight";i:0;}s:28:"views-editors_choice-block_2";a:4:{s:6:"module";s:5:"views";s:5:"delta";s:22:"editors_choice-block_2";s:6:"region";s:5:"right";s:6:"weight";i:1;}}}}', 0);

-- --------------------------------------------------------

--
-- Table structure for table `ctools_css_cache`
--

CREATE TABLE IF NOT EXISTS `ctools_css_cache` (
  `cid` varchar(128) NOT NULL,
  `filename` varchar(255) DEFAULT NULL,
  `css` longtext,
  `filter` tinyint(4) DEFAULT NULL,
  PRIMARY KEY (`cid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `ctools_css_cache`
--

INSERT INTO `ctools_css_cache` (`cid`, `filename`, `css`, `filter`) VALUES
('ctools-stylizer:_temporary:7d5dac5d92153e05b7fa2b919cb66925', 'sites/medicineandhealthukm.com/files/ctools/css/9ee5b43f08892ee550f191f22e7dedec.css', '/* Show only to IE7 */\n*:first-child+html .rounded-shadow-background,\n/* Show only to IE6 */\n* html .rounded-shadow-background  {\n  margin: 0 -10px 0 0 !important;\n  padding: 10px 0 5px 0 !important;\n}\n\n.rounded-shadow-top-edge, .rounded-shadow-bottom-edge, .rounded-shadow-left-edge, .rounded-shadow-right-edge, .rounded-shadow-wrap-corner {\n  position: relative;\n  /* hasLayout -1 ? For IE only */\n  zoom: 1;\n}\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 {\n  padding-top: 10px;\n  margin-bottom: 30px;\n  color: #000000;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-background {\n  margin: 10px;\n  background: #eee url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-background.png) repeat;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner {\n  margin: -10px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-top-edge {\n  top: -10px;\n  background: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-top-edge.png) repeat-x 0 top;\n  font-size: 1px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-bottom-edge {\n  bottom: -10px;\n  background: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-bottom-edge.png) repeat-x 0 bottom;\n  font-size: 1px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-left-edge {\n  background: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-left-edge.png) repeat-y 0 0;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-right-edge {\n  background: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-right-edge.png) repeat-y right 0;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-top-edge,\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-bottom-edge {\n  height: 19px;\n  margin: -10px 19px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-left,\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-right {\n  position: absolute;\n  top: 0;\n  height: 19px;\n  width: 19px;\n  margin: 0 -19px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-left {\n  background-image: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-top-left-corner.png);\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-right {\n  right: 0;\n  background-image: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-top-right-corner.png);\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-bottom-edge .rounded-shadow-left {\n  background-image: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-bottom-left-corner.png);\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-bottom-edge .rounded-shadow-right {\n  right: 0;\n  background-image: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-bottom-right-corner.png);\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .rounded-shadow-wrap-corner .rounded-shadow-right-edge {\n  padding: 3px 19px;\n}\n\n/*\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 div.admin-links {\n  margin-top: -19px;\n  margin-left: -12px;\n}\n\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 .panel-separator {\n  background: url(http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/ctools/style/_temporary/7d5dac5d92153e05b7fa2b919cb66925/rounded-shadow-bottom-edge.png) repeat-x 0 center;\n  font-size: 1px;\n  height: 30px;\n}\n*/\n.region-rounded-shadow-temporary-7d5dac5d92153e05b7fa2b919cb66925 {\n  font-family: Trebuchet MS, Trebuchet, Verdana, sans-serif;\n}\n', 0);

-- --------------------------------------------------------

--
-- Table structure for table `ctools_object_cache`
--

CREATE TABLE IF NOT EXISTS `ctools_object_cache` (
  `sid` varchar(64) NOT NULL,
  `name` varchar(128) NOT NULL,
  `obj` varchar(32) NOT NULL,
  `updated` int(10) unsigned NOT NULL DEFAULT '0',
  `data` longtext,
  PRIMARY KEY (`sid`,`obj`,`name`),
  KEY `updated` (`updated`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `dashboard_default`
--

CREATE TABLE IF NOT EXISTS `dashboard_default` (
  `id` smallint(6) NOT NULL AUTO_INCREMENT,
  `title` varchar(255) NOT NULL,
  `description` varchar(255) NOT NULL,
  `thumbnail` int(11) DEFAULT NULL,
  `tags` varchar(255) NOT NULL,
  `default_enabled` tinyint(4) NOT NULL,
  `widget_type` varchar(32) NOT NULL,
  `subtype` varchar(64) NOT NULL,
  `conf` longblob NOT NULL,
  PRIMARY KEY (`id`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=3 ;

--
-- Dumping data for table `dashboard_default`
--

INSERT INTO `dashboard_default` (`id`, `title`, `description`, `thumbnail`, `tags`, `default_enabled`, `widget_type`, `subtype`, `conf`) VALUES
(1, 'Recent Comments Block', 'Recent Comments.', 0, 'comments', 1, 'block', 'comment-0', ''),
(2, 'submission', 'Summary of paper status', 0, 'submission', 1, 'block', 'views-archivez-block', '');

-- --------------------------------------------------------

--
-- Table structure for table `dashboard_page`
--

CREATE TABLE IF NOT EXISTS `dashboard_page` (
  `page_id` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `uid` int(10) unsigned NOT NULL,
  `path` varchar(22) NOT NULL,
  `weight` tinyint(3) unsigned NOT NULL,
  `title` varchar(20) NOT NULL,
  PRIMARY KEY (`page_id`),
  UNIQUE KEY `uid_path` (`uid`,`path`),
  KEY `uid_weight` (`uid`,`weight`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=2 ;

--
-- Dumping data for table `dashboard_page`
--

INSERT INTO `dashboard_page` (`page_id`, `uid`, `path`, `weight`, `title`) VALUES
(1, 1, 'dashboard', 0, 'Dashboard');

-- --------------------------------------------------------

--
-- Table structure for table `dashboard_widget`
--

CREATE TABLE IF NOT EXISTS `dashboard_widget` (
  `widget_id` int(10) unsigned NOT NULL,
  `page_id` int(10) unsigned NOT NULL,
  `type` varchar(32) DEFAULT '',
  `subtype` varchar(64) DEFAULT '',
  `conf` longblob,
  `col` tinyint(3) unsigned NOT NULL,
  `weight` tinyint(3) unsigned NOT NULL,
  KEY `page_id_weight` (`page_id`,`weight`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `date_formats`
--

CREATE TABLE IF NOT EXISTS `date_formats` (
  `dfid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `format` varchar(100) CHARACTER SET utf8 COLLATE utf8_bin NOT NULL,
  `type` varchar(200) NOT NULL,
  `locked` tinyint(4) NOT NULL DEFAULT '0',
  PRIMARY KEY (`dfid`),
  UNIQUE KEY `formats` (`format`,`type`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=36 ;

--
-- Dumping data for table `date_formats`
--

INSERT INTO `date_formats` (`dfid`, `format`, `type`, `locked`) VALUES
(1, 'Y-m-d H:i', 'short', 1),
(2, 'm/d/Y - H:i', 'short', 1),
(3, 'd/m/Y - H:i', 'short', 1),
(4, 'Y/m/d - H:i', 'short', 1),
(5, 'd.m.Y - H:i', 'short', 1),
(6, 'm/d/Y - g:ia', 'short', 1),
(7, 'd/m/Y - g:ia', 'short', 1),
(8, 'Y/m/d - g:ia', 'short', 1),
(9, 'M j Y - H:i', 'short', 1),
(10, 'j M Y - H:i', 'short', 1),
(11, 'Y M j - H:i', 'short', 1),
(12, 'M j Y - g:ia', 'short', 1),
(13, 'j M Y - g:ia', 'short', 1),
(14, 'Y M j - g:ia', 'short', 1),
(15, 'D, Y-m-d H:i', 'medium', 1),
(16, 'D, m/d/Y - H:i', 'medium', 1),
(17, 'D, d/m/Y - H:i', 'medium', 1),
(18, 'D, Y/m/d - H:i', 'medium', 1),
(19, 'F j, Y - H:i', 'medium', 1),
(20, 'j F, Y - H:i', 'medium', 1),
(21, 'Y, F j - H:i', 'medium', 1),
(22, 'D, m/d/Y - g:ia', 'medium', 1),
(23, 'D, d/m/Y - g:ia', 'medium', 1),
(24, 'D, Y/m/d - g:ia', 'medium', 1),
(25, 'F j, Y - g:ia', 'medium', 1),
(26, 'j F Y - g:ia', 'medium', 1),
(27, 'Y, F j - g:ia', 'medium', 1),
(28, 'j. F Y - G:i', 'medium', 1),
(29, 'l, F j, Y - H:i', 'long', 1),
(30, 'l, j F, Y - H:i', 'long', 1),
(31, 'l, Y,  F j - H:i', 'long', 1),
(32, 'l, F j, Y - g:ia', 'long', 1),
(33, 'l, j F Y - g:ia', 'long', 1),
(34, 'l, Y,  F j - g:ia', 'long', 1),
(35, 'l, j. F Y - G:i', 'long', 1);

-- --------------------------------------------------------

--
-- Table structure for table `date_format_locale`
--

CREATE TABLE IF NOT EXISTS `date_format_locale` (
  `format` varchar(100) CHARACTER SET utf8 COLLATE utf8_bin NOT NULL,
  `type` varchar(200) NOT NULL,
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-- --------------------------------------------------------

--
-- Table structure for table `feeds_importer`
--

CREATE TABLE IF NOT EXISTS `feeds_importer` (
  `id` varchar(128) NOT NULL DEFAULT '',
  `config` text,
  PRIMARY KEY (`id`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `feeds_node_item`
--

CREATE TABLE IF NOT EXISTS `feeds_node_item` (
  `nid` int(10) unsigned NOT NULL,
  `id` varchar(128) NOT NULL DEFAULT '',
  `feed_nid` int(10) unsigned NOT NULL,
  `imported` int(11) NOT NULL DEFAULT '0',
  `url` text NOT NULL,
  `guid` text NOT NULL,
  `hash` varchar(32) NOT NULL DEFAULT '',
  PRIMARY KEY (`nid`),
  KEY `id` (`id`),
  KEY `feed_nid` (`feed_nid`),
  KEY `imported` (`imported`),
  KEY `url` (`url`(255)),
  KEY `guid` (`guid`(255))
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `feeds_push_subscriptions`
--

CREATE TABLE IF NOT EXISTS `feeds_push_subscriptions` (
  `domain` varchar(128) NOT NULL DEFAULT '',
  `subscriber_id` int(10) unsigned NOT NULL DEFAULT '0',
  `timestamp` int(11) NOT NULL DEFAULT '0',
  `hub` text NOT NULL,
  `topic` text NOT NULL,
  `secret` varchar(128) NOT NULL DEFAULT '',
  `status` varchar(64) NOT NULL DEFAULT '',
  `post_fields` text,
  PRIMARY KEY (`domain`,`subscriber_id`),
  KEY `timestamp` (`timestamp`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `feeds_source`
--

CREATE TABLE IF NOT EXISTS `feeds_source` (
  `id` varchar(128) NOT NULL DEFAULT '',
  `feed_nid` int(10) unsigned NOT NULL DEFAULT '0',
  `config` text,
  `source` text NOT NULL,
  `batch` longtext,
  PRIMARY KEY (`id`,`feed_nid`),
  KEY `id` (`id`),
  KEY `feed_nid` (`feed_nid`),
  KEY `id_source` (`id`,`source`(128))
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `feeds_term_item`
--

CREATE TABLE IF NOT EXISTS `feeds_term_item` (
  `tid` int(10) unsigned NOT NULL DEFAULT '0',
  `id` varchar(128) NOT NULL DEFAULT '',
  `feed_nid` int(10) unsigned NOT NULL,
  PRIMARY KEY (`tid`),
  KEY `id_feed_nid` (`id`,`feed_nid`),
  KEY `feed_nid` (`feed_nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `files`
--

CREATE TABLE IF NOT EXISTS `files` (
  `fid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `uid` int(10) unsigned NOT NULL DEFAULT '0',
  `filename` varchar(255) NOT NULL DEFAULT '',
  `filepath` varchar(255) NOT NULL DEFAULT '',
  `filemime` varchar(255) NOT NULL DEFAULT '',
  `filesize` int(10) unsigned NOT NULL DEFAULT '0',
  `status` int(11) NOT NULL DEFAULT '0',
  `timestamp` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`fid`),
  KEY `uid` (`uid`),
  KEY `status` (`status`),
  KEY `timestamp` (`timestamp`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=3224 ;

--
-- Dumping data for table `files`
--

INSERT INTO `files` (`fid`, `uid`, `filename`, `filepath`, `filemime`, `filesize`, `status`, `timestamp`) VALUES
(4, 1, 'Diagnostic dilemma.png', 'sites/default/files/Diagnostic dilemma.png', 'image/png', 70496, 1, 1321870903),
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(38, 1, '2008Vol3No1(Supplement)-cover.png', 'sites/default/files/manuscript/admin/2008Vol3No1(Supplement)-cover.png', 'image/png', 56896, 1, 1333614207),
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(381, 1, '2009Vol4No1-cover.jpg', 'sites/medicineandhealthukm.com/files/2009Vol4No1-cover.jpg', 'image/jpeg', 51147, 1, 1340596840),
(382, 1, '2009Vol4No2-cover.jpg', 'sites/medicineandhealthukm.com/files/2009Vol4No2-cover.jpg', 'image/jpeg', 65752, 1, 1340596863),
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(387, 1, 'cover.jpg', 'sites/medicineandhealthukm.com/files/cover.jpg', 'image/jpeg', 31403, 1, 1340936701),
(388, 1, 'medhealth.jpg', 'sites/medicineandhealthukm.com/files/medhealth.jpg', 'image/jpeg', 53775, 1, 1340945329),
(390, 1, '2006Vol1No1-cover.png', 'sites/medicineandhealthukm.com/files/2006Vol1No1-cover.png', 'image/png', 39470, 1, 1341217248),
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(392, 1, '2007Vol2No2-cover.png', 'sites/medicineandhealthukm.com/files/2007Vol2No2-cover.png', 'image/png', 48796, 1, 1341218208),
(393, 1, '2008Vol3No1-cover.png', 'sites/medicineandhealthukm.com/files/2008Vol3No1-cover.png', 'image/png', 54171, 1, 1341218553),
(394, 1, '2008Vol3No1(Supplement)-cover.png', 'sites/medicineandhealthukm.com/files/2008Vol3No1(Supplement)-cover.png', 'image/png', 56896, 1, 1341218606),
(395, 1, '2008Vol3No2-cover.png', 'sites/medicineandhealthukm.com/files/2008Vol3No2-cover.png', 'image/png', 62091, 1, 1341219390),
(396, 1, '2011Vol6No1Sup.jpg', 'sites/medicineandhealthukm.com/files/2011Vol6No1Sup.jpg', 'image/jpeg', 49950, 1, 1341219506),
(397, 1, '07-MS 128 (107-113)a.jpg', 'sites/medicineandhealthukm.com/files/manuscript/admin/07-MS 128 (107-113)a.jpg', 'image/jpeg', 27554, 1, 1341228606),
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-- --------------------------------------------------------

--
-- Table structure for table `filters`
--

CREATE TABLE IF NOT EXISTS `filters` (
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--
-- Dumping data for table `filters`
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-- --------------------------------------------------------

--
-- Table structure for table `filter_formats`
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--
-- Dumping data for table `filter_formats`
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-- --------------------------------------------------------

--
-- Table structure for table `flags`
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--
-- Dumping data for table `flags`
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-- --------------------------------------------------------

--
-- Table structure for table `flag_actions`
--

CREATE TABLE IF NOT EXISTS `flag_actions` (
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-- --------------------------------------------------------

--
-- Table structure for table `flag_content`
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--
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-- --------------------------------------------------------

--
-- Table structure for table `flag_counts`
--

CREATE TABLE IF NOT EXISTS `flag_counts` (
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--
-- Dumping data for table `flag_counts`
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-- --------------------------------------------------------

--
-- Table structure for table `flag_types`
--

CREATE TABLE IF NOT EXISTS `flag_types` (
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--
-- Dumping data for table `flag_types`
--

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-- --------------------------------------------------------

--
-- Table structure for table `flood`
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--
-- Dumping data for table `flood`
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-- --------------------------------------------------------

--
-- Table structure for table `history`
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--
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-- --------------------------------------------------------

--
-- Table structure for table `imagecache_action`
--

CREATE TABLE IF NOT EXISTS `imagecache_action` (
  `actionid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `presetid` int(10) unsigned NOT NULL DEFAULT '0',
  `weight` int(11) NOT NULL DEFAULT '0',
  `module` varchar(255) NOT NULL,
  `action` varchar(255) NOT NULL,
  `data` longtext NOT NULL,
  PRIMARY KEY (`actionid`),
  KEY `presetid` (`presetid`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=8 ;

--
-- Dumping data for table `imagecache_action`
--

INSERT INTO `imagecache_action` (`actionid`, `presetid`, `weight`, `module`, `action`, `data`) VALUES
(1, 1, 0, 'imagecrop', 'imagecrop_javascript', 'a:6:{s:5:"width";s:2:"70";s:6:"height";s:2:"70";s:7:"xoffset";s:6:"center";s:7:"yoffset";s:6:"center";s:9:"resizable";i:1;s:18:"disable_if_no_data";i:1;}'),
(2, 1, 0, 'imagecache', 'imagecache_scale', 'a:3:{s:5:"width";s:2:"70";s:6:"height";s:0:"";s:7:"upscale";i:1;}'),
(3, 2, -1, 'imagecrop', 'imagecrop_javascript', 'a:6:{s:5:"width";s:3:"400";s:6:"height";s:3:"200";s:7:"xoffset";s:6:"center";s:7:"yoffset";s:6:"center";s:9:"resizable";i:1;s:18:"disable_if_no_data";i:1;}'),
(4, 2, 0, 'imagecache', 'imagecache_scale', 'a:3:{s:5:"width";s:3:"400";s:6:"height";s:0:"";s:7:"upscale";i:1;}'),
(5, 3, 0, 'imagecache', 'imagecache_scale_and_crop', 'a:2:{s:5:"width";s:2:"75";s:6:"height";s:2:"75";}'),
(6, 4, 0, 'imagecache', 'imagecache_resize', 'a:2:{s:5:"width";s:3:"120";s:6:"height";s:3:"168";}'),
(7, 5, 0, 'imagecache', 'imagecache_scale_and_crop', 'a:2:{s:5:"width";s:3:"200";s:6:"height";s:3:"245";}');

-- --------------------------------------------------------

--
-- Table structure for table `imagecache_preset`
--

CREATE TABLE IF NOT EXISTS `imagecache_preset` (
  `presetid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `presetname` varchar(255) NOT NULL,
  PRIMARY KEY (`presetid`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=6 ;

--
-- Dumping data for table `imagecache_preset`
--

INSERT INTO `imagecache_preset` (`presetid`, `presetname`) VALUES
(1, 'author_photo'),
(2, '400xY'),
(3, 'jcarousel'),
(4, 'issue'),
(5, 'slide');

-- --------------------------------------------------------

--
-- Table structure for table `imagecrop`
--

CREATE TABLE IF NOT EXISTS `imagecrop` (
  `fid` int(10) unsigned NOT NULL,
  `presetid` int(10) unsigned NOT NULL,
  `reference` varchar(40) NOT NULL DEFAULT '',
  `xoffset` int(10) unsigned NOT NULL,
  `yoffset` int(10) unsigned NOT NULL,
  `width` int(10) unsigned NOT NULL,
  `height` int(10) unsigned NOT NULL,
  `scale` varchar(10) NOT NULL DEFAULT 'original',
  KEY `fid` (`fid`),
  KEY `presetid` (`presetid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `imagecrop`
--

INSERT INTO `imagecrop` (`fid`, `presetid`, `reference`, `xoffset`, `yoffset`, `width`, `height`, `scale`) VALUES
(2062, 1, 'files', 0, 0, 70, 70, 'original'),
(2928, 1, 'files', 46, 20, 270, 315, 'original');

-- --------------------------------------------------------

--
-- Table structure for table `imce_files`
--

CREATE TABLE IF NOT EXISTS `imce_files` (
  `fid` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`fid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `imce_files`
--

INSERT INTO `imce_files` (`fid`) VALUES
(453),
(454),
(455);

-- --------------------------------------------------------

--
-- Table structure for table `job_schedule`
--

CREATE TABLE IF NOT EXISTS `job_schedule` (
  `callback` varchar(128) NOT NULL DEFAULT '',
  `type` varchar(128) NOT NULL DEFAULT '',
  `id` int(10) unsigned NOT NULL DEFAULT '0',
  `last` int(10) unsigned NOT NULL DEFAULT '0',
  `period` int(10) unsigned NOT NULL DEFAULT '0',
  `next` int(10) unsigned NOT NULL DEFAULT '0',
  `periodic` smallint(5) unsigned NOT NULL DEFAULT '0',
  `scheduled` int(10) unsigned NOT NULL DEFAULT '0',
  KEY `callback_type_id` (`callback`,`type`,`id`),
  KEY `callback_type` (`callback`,`type`),
  KEY `next` (`next`),
  KEY `scheduled` (`scheduled`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `l10n_update_file`
--

CREATE TABLE IF NOT EXISTS `l10n_update_file` (
  `project` varchar(50) NOT NULL,
  `language` varchar(12) NOT NULL,
  `type` varchar(50) NOT NULL DEFAULT '',
  `filename` varchar(255) NOT NULL DEFAULT '',
  `fileurl` varchar(255) NOT NULL DEFAULT '',
  `filepath` varchar(255) NOT NULL DEFAULT '',
  `timestamp` int(11) DEFAULT '0',
  `version` varchar(128) NOT NULL DEFAULT '',
  `status` int(11) NOT NULL DEFAULT '1',
  `last_updated` int(11) DEFAULT '0',
  `last_checked` int(11) DEFAULT '0',
  `import_date` int(11) DEFAULT '0',
  PRIMARY KEY (`project`,`language`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `l10n_update_project`
--

CREATE TABLE IF NOT EXISTS `l10n_update_project` (
  `name` varchar(50) NOT NULL,
  `project_type` varchar(50) NOT NULL,
  `core` varchar(128) NOT NULL DEFAULT '',
  `version` varchar(128) NOT NULL DEFAULT '',
  `l10n_server` varchar(255) NOT NULL DEFAULT '',
  `l10n_path` varchar(255) NOT NULL DEFAULT '',
  `status` int(11) NOT NULL DEFAULT '1',
  PRIMARY KEY (`name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `l10n_update_project`
--

INSERT INTO `l10n_update_project` (`name`, `project_type`, `core`, `version`, `l10n_server`, `l10n_path`, `status`) VALUES
('accordion_menu', 'module', '6.x', '6.x-1.2+1-dev', '', 'http://ftp.drupal.org/files/translations/6.x/accordion_menu/accordion_menu-6.x-1.2+1-dev.%language.po', 1),
('admin', 'module', '6.x', '6.x-2.0', '', 'http://ftp.drupal.org/files/translations/6.x/admin/admin-6.x-2.0.%language.po', 1),
('admin_theme', 'module', '6.x', '6.x-1.3', '', 'http://ftp.drupal.org/files/translations/6.x/admin_theme/admin_theme-6.x-1.3.%language.po', 1),
('advanced_help', 'module', '6.x', '6.x-1.2', '', 'http://ftp.drupal.org/files/translations/6.x/advanced_help/advanced_help-6.x-1.2.%language.po', 1),
('advuser', 'module', '6.x', '6.x-2.3', '', 'http://ftp.drupal.org/files/translations/6.x/advuser/advuser-6.x-2.3.%language.po', 1),
('apply_for_role', 'module', '6.x', '6.x-1.9', '', 'http://ftp.drupal.org/files/translations/6.x/apply_for_role/apply_for_role-6.x-1.9.%language.po', 1),
('autoassignrole', 'module', '6.x', '6.x-1.2', '', 'http://ftp.drupal.org/files/translations/6.x/autoassignrole/autoassignrole-6.x-1.2.%language.po', 1),
('autoload', 'module', '6.x', '6.x-1.4', '', 'http://ftp.drupal.org/files/translations/6.x/autoload/autoload-6.x-1.4.%language.po', 1),
('auto_nodetitle', 'module', '6.x', '6.x-1.x-dev', '', 'http://ftp.drupal.org/files/translations/6.x/auto_nodetitle/auto_nodetitle-6.x-1.x-dev.%language.po', 1),
('backup_migrate', 'module', '6.x', '6.x-2.2', '', 'http://ftp.drupal.org/files/translations/6.x/backup_migrate/backup_migrate-6.x-2.2.%language.po', 1),
('casetracker', 'module', '6.x', '6.x-1.0-beta9', '', 'http://ftp.drupal.org/files/translations/6.x/casetracker/casetracker-6.x-1.0-beta9.%language.po', 1),
('cck', 'module', '6.x', '6.x-2.9', '', 'http://ftp.drupal.org/files/translations/6.x/cck/cck-6.x-2.9.%language.po', 1),
('cck_tweak_button', 'module', '6.x', '6.x-0.1-dev', '', 'http://ftp.drupal.org/files/translations/6.x/cck_tweak_button/cck_tweak_button-6.x-0.1-dev.%language.po', 1),
('ckeditor', 'module', '6.x', '6.x-1.2', '', 'http://ftp.drupal.org/files/translations/6.x/ckeditor/ckeditor-6.x-1.2.%language.po', 1),
('cmf', 'module', '6.x', '6.x-2.01', '', 'http://ftp.drupal.org/files/translations/6.x/cmf/cmf-6.x-2.01.%language.po', 0),
('conference', 'module', '6.x', '6.x-1.x-dev', '', 'http://ftp.drupal.org/files/translations/6.x/conference/conference-6.x-1.x-dev.%language.po', 1),
('content_profile', 'module', '6.x', '6.x-1.0', '', 'http://ftp.drupal.org/files/translations/6.x/content_profile/content_profile-6.x-1.0.%language.po', 1),
('context', 'module', '6.x', '6.x-3.0', '', 'http://ftp.drupal.org/files/translations/6.x/context/context-6.x-3.0.%language.po', 1),
('ctools', 'module', '6.x', '6.x-1.8', '', 'http://ftp.drupal.org/files/translations/6.x/ctools/ctools-6.x-1.8.%language.po', 1),
('custompage', 'module', '6.x', '6.x-1.17', '', 'http://ftp.drupal.org/files/translations/6.x/custompage/custompage-6.x-1.17.%language.po', 1),
('dashboard', 'module', '6.x', '6.x-2.1', '', 'http://ftp.drupal.org/files/translations/6.x/dashboard/dashboard-6.x-2.1.%language.po', 1),
('date', 'module', '6.x', '6.x-2.7', '', 'http://ftp.drupal.org/files/translations/6.x/date/date-6.x-2.7.%language.po', 1),
('devel', 'module', '6.x', '6.x-1.23', '', 'http://ftp.drupal.org/files/translations/6.x/devel/devel-6.x-1.23.%language.po', 1),
('diff', 'module', '6.x', '6.x-2.1', '', 'http://ftp.drupal.org/files/translations/6.x/diff/diff-6.x-2.1.%language.po', 1),
('distro', 'module', '6.x', '6.x-1.0-rc1', '', 'http://ftp.drupal.org/files/translations/6.x/distro/distro-6.x-1.0-rc1.%language.po', 1),
('document_cloud', 'module', '6.x', '6.x-1.5', '', 'http://ftp.drupal.org/files/translations/6.x/document_cloud/document_cloud-6.x-1.5.%language.po', 1),
('download_count', 'module', '6.x', '6.x-2.0+8-dev', '', 'http://ftp.drupal.org/files/translations/6.x/download_count/download_count-6.x-2.0+8-dev.%language.po', 1),
('drupal', 'core', '6.x', '6.20', '', 'http://ftp.drupal.org/files/translations/6.x/drupal/drupal-6.20.%language.po', 1),
('email', 'module', '6.x', '6.x-1.3', '', 'http://ftp.drupal.org/files/translations/6.x/email/email-6.x-1.3.%language.po', 1),
('emfield', 'module', '6.x', '6.x-1.26', '', 'http://ftp.drupal.org/files/translations/6.x/emfield/emfield-6.x-1.26.%language.po', 1),
('features', 'module', '6.x', '6.x-1.0', '', 'http://ftp.drupal.org/files/translations/6.x/features/features-6.x-1.0.%language.po', 1),
('feeds', 'module', '6.x', '6.x-1.0-beta10', '', 'http://ftp.drupal.org/files/translations/6.x/feeds/feeds-6.x-1.0-beta10.%language.po', 1),
('filefield', 'module', '6.x', '6.x-3.9', '', 'http://ftp.drupal.org/files/translations/6.x/filefield/filefield-6.x-3.9.%language.po', 1),
('filefield_sources', 'module', '6.x', '6.x-1.2', '', 'http://ftp.drupal.org/files/translations/6.x/filefield_sources/filefield_sources-6.x-1.2.%language.po', 1),
('flag', 'module', '6.x', '6.x-1.3', '', 'http://ftp.drupal.org/files/translations/6.x/flag/flag-6.x-1.3.%language.po', 1),
('flickrapi', 'module', '6.x', '6.x-1.1', '', 'http://ftp.drupal.org/files/translations/6.x/flickrapi/flickrapi-6.x-1.1.%language.po', 1),
('gmap', 'module', '6.x', '6.x-1.1', '', 'http://ftp.drupal.org/files/translations/6.x/gmap/gmap-6.x-1.1.%language.po', 1),
('ie_css_optimizer', 'module', '6.x', '6.x-1.1', '', 'http://ftp.drupal.org/files/translations/6.x/ie_css_optimizer/ie_css_optimizer-6.x-1.1.%language.po', 1),
('imageapi', 'module', '6.x', '6.x-1.9', '', 'http://ftp.drupal.org/files/translations/6.x/imageapi/imageapi-6.x-1.9.%language.po', 1),
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-- --------------------------------------------------------

--
-- Table structure for table `languages`
--

CREATE TABLE IF NOT EXISTS `languages` (
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--
-- Dumping data for table `languages`
--

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-- --------------------------------------------------------

--
-- Table structure for table `locales_source`
--

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-- --------------------------------------------------------

--
-- Table structure for table `locales_target`
--

CREATE TABLE IF NOT EXISTS `locales_target` (
  `lid` int(11) NOT NULL DEFAULT '0',
  `translation` blob NOT NULL,
  `language` varchar(12) NOT NULL DEFAULT '',
  `plid` int(11) NOT NULL DEFAULT '0',
  `plural` int(11) NOT NULL DEFAULT '0',
  `l10n_status` int(11) NOT NULL DEFAULT '0',
  PRIMARY KEY (`language`,`lid`,`plural`),
  KEY `lid` (`lid`),
  KEY `plid` (`plid`),
  KEY `plural` (`plural`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `menu_custom`
--

CREATE TABLE IF NOT EXISTS `menu_custom` (
  `menu_name` varchar(32) NOT NULL DEFAULT '',
  `title` varchar(255) NOT NULL DEFAULT '',
  `description` text,
  PRIMARY KEY (`menu_name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `menu_custom`
--

INSERT INTO `menu_custom` (`menu_name`, `title`, `description`) VALUES
('admin', 'Admin', 'Administrative links.'),
('devel', 'Development', 'Development links.'),
('features', 'Features', 'Menu items for any enabled features.'),
('menu-accord', 'Main Menu', ''),
('menu-account', 'My Account', ''),
('menu-footer-primary', 'Footer Primary', ''),
('menu-footer-secondary', 'Footer Secondary', ''),
('menu-top-menu', 'Top Menu', ''),
('navigation', 'Navigation', 'The navigation menu is provided by Drupal and is the main interactive menu for any site. It is usually the only menu that contains personalized links for authenticated users, and is often not even visible to anonymous users.'),
('primary-links', 'Primary links', 'Primary links are often used at the theme layer to show the major sections of a site. A typical representation for primary links would be tabs along the top.'),
('secondary-links', 'Secondary links', 'Secondary links are often used for pages like legal notices, contact details, and other secondary navigation items that play a lesser role than primary links');

-- --------------------------------------------------------

--
-- Table structure for table `menu_links`
--

CREATE TABLE IF NOT EXISTS `menu_links` (
  `menu_name` varchar(32) NOT NULL DEFAULT '',
  `mlid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `plid` int(10) unsigned NOT NULL DEFAULT '0',
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  `router_path` varchar(255) NOT NULL DEFAULT '',
  `link_title` varchar(255) NOT NULL DEFAULT '',
  `options` text,
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  `has_children` smallint(6) NOT NULL DEFAULT '0',
  `expanded` smallint(6) NOT NULL DEFAULT '0',
  `weight` int(11) NOT NULL DEFAULT '0',
  `depth` smallint(6) NOT NULL DEFAULT '0',
  `customized` smallint(6) NOT NULL DEFAULT '0',
  `p1` int(10) unsigned NOT NULL DEFAULT '0',
  `p2` int(10) unsigned NOT NULL DEFAULT '0',
  `p3` int(10) unsigned NOT NULL DEFAULT '0',
  `p4` int(10) unsigned NOT NULL DEFAULT '0',
  `p5` int(10) unsigned NOT NULL DEFAULT '0',
  `p6` int(10) unsigned NOT NULL DEFAULT '0',
  `p7` int(10) unsigned NOT NULL DEFAULT '0',
  `p8` int(10) unsigned NOT NULL DEFAULT '0',
  `p9` int(10) unsigned NOT NULL DEFAULT '0',
  `updated` smallint(6) NOT NULL DEFAULT '0',
  PRIMARY KEY (`mlid`),
  KEY `path_menu` (`link_path`(128),`menu_name`),
  KEY `menu_plid_expand_child` (`menu_name`,`plid`,`expanded`,`has_children`),
  KEY `menu_parents` (`menu_name`,`p1`,`p2`,`p3`,`p4`,`p5`,`p6`,`p7`,`p8`,`p9`),
  KEY `router_path` (`router_path`(128))
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=1021 ;

--
-- Dumping data for table `menu_links`
--

INSERT INTO `menu_links` (`menu_name`, `mlid`, `plid`, `link_path`, `router_path`, `link_title`, `options`, `module`, `hidden`, `external`, `has_children`, `expanded`, `weight`, `depth`, `customized`, `p1`, `p2`, `p3`, `p4`, `p5`, `p6`, `p7`, `p8`, `p9`, `updated`) VALUES
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('admin', 138, 124, 'admin/settings/uploads', 'admin/settings/uploads', 'File uploads', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:45:"Control how files may be attached to content.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 138, 0, 0, 0, 0, 0, 0, 0),
('admin', 139, 124, 'admin/settings/image-toolkit', 'admin/settings/image-toolkit', 'Image toolkit', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:74:"Choose which image toolkit to use if you have installed optional toolkits.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 139, 0, 0, 0, 0, 0, 0, 0),
('admin', 140, 124, 'admin/settings/filters', 'admin/settings/filters', 'Input formats', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:127:"Configure how content input by users is filtered, including allowed HTML tags. Also allows enabling of module-provided filters.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 140, 0, 0, 0, 0, 0, 0, 0),
('admin', 141, 124, 'admin/settings/language', 'admin/settings/language', 'Languages', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:55:"Configure languages for content and the user interface.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 141, 0, 0, 0, 0, 0, 0, 0),
('admin', 142, 124, 'admin/settings/logging', 'admin/settings/logging', 'Logging and alerts', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:156:"Settings for logging and alerts modules. Various modules can route Drupal''s system events to different destination, such as syslog, database, email, ...etc.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 142, 0, 0, 0, 0, 0, 0, 0),
('admin', 143, 123, 'admin/build/menu', 'admin/build/menu', 'Menus', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:116:"Control your site''s navigation menu, primary links and secondary links. as well as rename and reorganize menu items.";}}', 'system', 0, 0, 1, 0, 0, 3, 0, 114, 123, 143, 0, 0, 0, 0, 0, 0, 0),
('admin', 144, 123, 'admin/build/modules', 'admin/build/modules', 'Modules', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 123, 144, 0, 0, 0, 0, 0, 0, 0),
('admin', 145, 124, 'admin/settings/performance', 'admin/settings/performance', 'Performance', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:101:"Enable or disable page caching for anonymous users and set CSS and JS bandwidth optimization options.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 145, 0, 0, 0, 0, 0, 0, 0),
('admin', 146, 125, 'admin/user/permissions', 'admin/user/permissions', 'Permissions', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:64:"Determine access to features by selecting permissions for roles.";}}', 'system', 0, 0, 0, 0, -45, 3, 1, 114, 125, 146, 0, 0, 0, 0, 0, 0, 0),
('admin', 147, 119, 'admin/content/node-settings', 'admin/content/node-settings', 'Post settings', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:126:"Control posting behavior, such as teaser length, requiring previews before posting, and the number of posts on the front page.";}}', 'system', 0, 0, 0, 0, -40, 3, 1, 114, 119, 147, 0, 0, 0, 0, 0, 0, 0),
('admin', 148, 125, 'admin/user/profile', 'admin/user/profile', 'Profiles', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:42:"Create customizable fields for your users.";}}', 'system', 0, 0, 0, 0, -43, 3, 1, 114, 125, 148, 0, 0, 0, 0, 0, 0, 0),
('admin', 149, 119, 'admin/content/rss-publishing', 'admin/content/rss-publishing', 'RSS publishing', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:92:"Configure the number of items per feed and whether feeds should be titles/teasers/full-text.";}}', 'system', 0, 0, 0, 0, -38, 3, 1, 114, 119, 149, 0, 0, 0, 0, 0, 0, 0),
('admin', 150, 122, 'admin/reports/hits', 'admin/reports/hits', 'Recent hits', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:43:"View pages that have recently been visited.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 122, 150, 0, 0, 0, 0, 0, 0, 0),
('admin', 151, 125, 'admin/user/roles', 'admin/user/roles', 'Roles', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, -42, 3, 1, 114, 125, 151, 0, 0, 0, 0, 0, 0, 0),
('admin', 152, 124, 'admin/settings/search', 'admin/settings/search', 'Search settings', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:66:"Configure relevance settings for search and other indexing options";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 152, 0, 0, 0, 0, 0, 0, 0),
('admin', 153, 124, 'admin/settings/site-information', 'admin/settings/site-information', 'Site information', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:107:"Change basic site information, such as the site name, slogan, e-mail address, mission, front page and more.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 153, 0, 0, 0, 0, 0, 0, 0),
('admin', 154, 124, 'admin/settings/site-maintenance', 'admin/settings/site-maintenance', 'Site maintenance', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:63:"Take the site off-line for maintenance or bring it back online.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 154, 0, 0, 0, 0, 0, 0, 0),
('admin', 155, 122, 'admin/reports/status', 'admin/reports/status', 'Status report', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, 10, 3, 0, 114, 122, 155, 0, 0, 0, 0, 0, 0, 0),
('admin', 156, 119, 'admin/content/taxonomy', 'admin/content/taxonomy', 'Taxonomy', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, -37, 3, 1, 114, 119, 156, 0, 0, 0, 0, 0, 0, 0),
('navigation', 157, 0, 'taxonomy/term/%', 'taxonomy/term/%', 'Taxonomy term', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 157, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 158, 123, 'admin/build/themes', 'admin/build/themes', 'Themes', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:57:"Change which theme your site uses or allows users to set.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 123, 158, 0, 0, 0, 0, 0, 0, 0),
('admin', 159, 122, 'admin/reports/pages', 'admin/reports/pages', 'Top pages', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:41:"View pages that have been hit frequently.";}}', 'system', 0, 0, 0, 0, 1, 3, 0, 114, 122, 159, 0, 0, 0, 0, 0, 0, 0),
('admin', 160, 122, 'admin/reports/referrers', 'admin/reports/referrers', 'Top referrers', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:19:"View top referrers.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 122, 160, 0, 0, 0, 0, 0, 0, 0),
('admin', 161, 122, 'admin/reports/search', 'admin/reports/search', 'Top search phrases', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:33:"View most popular search phrases.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 122, 161, 0, 0, 0, 0, 0, 0, 0),
('admin', 162, 122, 'admin/reports/visitors', 'admin/reports/visitors', 'Top visitors', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:34:"View visitors that hit many pages.";}}', 'system', 0, 0, 0, 0, 2, 3, 0, 114, 122, 162, 0, 0, 0, 0, 0, 0, 0),
('admin', 163, 123, 'admin/build/translate', 'admin/build/translate', 'Translate interface', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:59:"Translate the built in interface and optionally other text.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 123, 163, 0, 0, 0, 0, 0, 0, 0),
('admin', 164, 123, 'admin/build/path', 'admin/build/path', 'URL aliases', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:46:"Change your site''s URL paths by aliasing them.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 123, 164, 0, 0, 0, 0, 0, 0, 0),
('admin', 165, 125, 'admin/user/settings', 'admin/user/settings', 'User settings', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:101:"Configure default behavior of users, including registration requirements, e-mails, and user pictures.";}}', 'system', 0, 0, 0, 0, -40, 3, 1, 114, 125, 165, 0, 0, 0, 0, 0, 0, 0),
('admin', 166, 125, 'admin/user/user', 'admin/user/user', 'Users', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:26:"List, add, and edit users.";}}', 'system', 0, 0, 0, 0, -39, 3, 1, 114, 125, 166, 0, 0, 0, 0, 0, 0, 0),
('admin', 167, 120, 'admin/help/block', 'admin/help/block', 'block', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 167, 0, 0, 0, 0, 0, 0, 0),
('admin', 168, 120, 'admin/help/blog', 'admin/help/blog', 'blog', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 168, 0, 0, 0, 0, 0, 0, 0),
('admin', 169, 120, 'admin/help/comment', 'admin/help/comment', 'comment', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 169, 0, 0, 0, 0, 0, 0, 0),
('admin', 170, 120, 'admin/help/filter', 'admin/help/filter', 'filter', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 170, 0, 0, 0, 0, 0, 0, 0),
('admin', 171, 120, 'admin/help/help', 'admin/help/help', 'help', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 171, 0, 0, 0, 0, 0, 0, 0),
('admin', 172, 120, 'admin/help/locale', 'admin/help/locale', 'locale', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 172, 0, 0, 0, 0, 0, 0, 0),
('admin', 173, 120, 'admin/help/menu', 'admin/help/menu', 'menu', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 173, 0, 0, 0, 0, 0, 0, 0),
('admin', 174, 120, 'admin/help/node', 'admin/help/node', 'node', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 174, 0, 0, 0, 0, 0, 0, 0),
('admin', 176, 120, 'admin/help/path', 'admin/help/path', 'path', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 176, 0, 0, 0, 0, 0, 0, 0),
('admin', 177, 120, 'admin/help/profile', 'admin/help/profile', 'profile', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 177, 0, 0, 0, 0, 0, 0, 0),
('admin', 178, 120, 'admin/help/search', 'admin/help/search', 'search', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 178, 0, 0, 0, 0, 0, 0, 0),
('admin', 179, 120, 'admin/help/statistics', 'admin/help/statistics', 'statistics', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 179, 0, 0, 0, 0, 0, 0, 0),
('admin', 180, 120, 'admin/help/system', 'admin/help/system', 'system', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 180, 0, 0, 0, 0, 0, 0, 0),
('admin', 181, 120, 'admin/help/taxonomy', 'admin/help/taxonomy', 'taxonomy', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 181, 0, 0, 0, 0, 0, 0, 0),
('admin', 182, 120, 'admin/help/translation', 'admin/help/translation', 'translation', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 182, 0, 0, 0, 0, 0, 0, 0),
('admin', 183, 120, 'admin/help/upload', 'admin/help/upload', 'upload', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 183, 0, 0, 0, 0, 0, 0, 0),
('admin', 184, 120, 'admin/help/user', 'admin/help/user', 'user', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 184, 0, 0, 0, 0, 0, 0, 0),
('admin', 185, 140, 'admin/settings/filters/%', 'admin/settings/filters/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 140, 185, 0, 0, 0, 0, 0, 0),
('admin', 186, 148, 'admin/user/profile/add', 'admin/user/profile/add', 'Add field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 148, 186, 0, 0, 0, 0, 0, 0),
('admin', 187, 119, 'admin/content/node-type/blog', 'admin/content/node-type/blog', 'Blog entry', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 119, 187, 0, 0, 0, 0, 0, 0, 0),
('admin', 188, 131, 'admin/settings/clean-urls/check', 'admin/settings/clean-urls/check', 'Clean URL check', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 131, 188, 0, 0, 0, 0, 0, 0),
('admin', 189, 152, 'admin/settings/search/wipe', 'admin/settings/search/wipe', 'Clear index', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 152, 189, 0, 0, 0, 0, 0, 0),
('admin', 190, 128, 'admin/settings/actions/configure', 'admin/settings/actions/configure', 'Configure an advanced action', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 128, 190, 0, 0, 0, 0, 0, 0),
('admin', 191, 130, 'admin/build/block/configure', 'admin/build/block/configure', 'Configure block', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 130, 191, 0, 0, 0, 0, 0, 0),
('admin', 192, 123, 'admin/build/menu-customize/%', 'admin/build/menu-customize/%', 'Customize menu', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 123, 192, 0, 0, 0, 0, 0, 0, 0),
('admin', 193, 135, 'admin/settings/date-time/lookup', 'admin/settings/date-time/lookup', 'Date and time lookup', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 135, 193, 0, 0, 0, 0, 0, 0),
('admin', 195, 164, 'admin/build/path/delete', 'admin/build/path/delete', 'Delete alias', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 164, 195, 0, 0, 0, 0, 0, 0),
('admin', 196, 130, 'admin/build/block/delete', 'admin/build/block/delete', 'Delete block', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 130, 196, 0, 0, 0, 0, 0, 0),
('admin', 197, 148, 'admin/user/profile/delete', 'admin/user/profile/delete', 'Delete field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 148, 197, 0, 0, 0, 0, 0, 0),
('admin', 198, 140, 'admin/settings/filters/delete', 'admin/settings/filters/delete', 'Delete input format', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 140, 198, 0, 0, 0, 0, 0, 0),
('admin', 199, 127, 'admin/user/rules/delete', 'admin/user/rules/delete', 'Delete rule', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 127, 199, 0, 0, 0, 0, 0, 0),
('admin', 200, 122, 'admin/reports/access/%', 'admin/reports/access/%', 'Details', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:16:"View access log.";}}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 122, 200, 0, 0, 0, 0, 0, 0, 0),
('admin', 201, 164, 'admin/build/path/edit', 'admin/build/path/edit', 'Edit alias', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 164, 201, 0, 0, 0, 0, 0, 0),
('admin', 202, 148, 'admin/user/profile/edit', 'admin/user/profile/edit', 'Edit field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 148, 202, 0, 0, 0, 0, 0, 0),
('admin', 203, 151, 'admin/user/roles/edit', 'admin/user/roles/edit', 'Edit role', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 151, 203, 0, 0, 0, 0, 0, 0),
('admin', 204, 127, 'admin/user/rules/edit', 'admin/user/rules/edit', 'Edit rule', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 127, 204, 0, 0, 0, 0, 0, 0),
('admin', 205, 156, 'admin/content/taxonomy/%', 'admin/content/taxonomy/%', 'List terms', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 119, 156, 205, 0, 0, 0, 0, 0, 0),
('admin', 206, 155, 'admin/reports/status/php', 'admin/reports/status/php', 'PHP', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 122, 155, 206, 0, 0, 0, 0, 0, 0),
('admin', 207, 148, 'admin/user/profile/autocomplete', 'admin/user/profile/autocomplete', 'Profile category autocomplete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 125, 148, 207, 0, 0, 0, 0, 0, 0),
('admin', 208, 147, 'admin/content/node-settings/rebuild', 'admin/content/node-settings/rebuild', 'Rebuild permissions', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 119, 147, 208, 0, 0, 0, 0, 0, 0),
('admin', 209, 128, 'admin/settings/actions/orphan', 'admin/settings/actions/orphan', 'Remove orphans', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 128, 209, 0, 0, 0, 0, 0, 0),
('admin', 210, 155, 'admin/reports/status/run-cron', 'admin/reports/status/run-cron', 'Run cron', 'a:1:{s:5:"alter";b:1;}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 122, 155, 210, 0, 0, 0, 0, 0, 0),
('admin', 211, 155, 'admin/reports/status/sql', 'admin/reports/status/sql', 'SQL', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 122, 155, 211, 0, 0, 0, 0, 0, 0),
('admin', 212, 141, 'admin/settings/language/delete/%', 'admin/settings/language/delete/%', 'Confirm', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 141, 212, 0, 0, 0, 0, 0, 0),
('admin', 213, 0, 'admin/content/node-type/blog/delete', 'admin/content/node-type/blog/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 213, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 214, 128, 'admin/settings/actions/delete/%', 'admin/settings/actions/delete/%', 'Delete action', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:17:"Delete an action.";}}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 128, 214, 0, 0, 0, 0, 0, 0),
('admin', 215, 0, 'admin/build/menu-customize/%/delete', 'admin/build/menu-customize/%/delete', 'Delete menu', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 215, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 216, 163, 'admin/build/translate/delete/%', 'admin/build/translate/delete/%', 'Delete string', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 163, 216, 0, 0, 0, 0, 0, 0),
('admin', 217, 141, 'admin/settings/language/edit/%', 'admin/settings/language/edit/%', 'Edit language', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 141, 217, 0, 0, 0, 0, 0, 0),
('admin', 218, 163, 'admin/build/translate/edit/%', 'admin/build/translate/edit/%', 'Edit string', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 163, 218, 0, 0, 0, 0, 0, 0),
('admin', 219, 156, 'admin/content/taxonomy/edit/term', 'admin/content/taxonomy/edit/term', 'Edit term', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 119, 156, 219, 0, 0, 0, 0, 0, 0),
('admin', 220, 130, 'admin/build/block/list/js', 'admin/build/block/list/js', 'JavaScript List Form', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 130, 220, 0, 0, 0, 0, 0, 0),
('admin', 221, 144, 'admin/build/modules/list/confirm', 'admin/build/modules/list/confirm', 'List', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 144, 221, 0, 0, 0, 0, 0, 0),
('admin', 222, 144, 'admin/build/modules/uninstall/confirm', 'admin/build/modules/uninstall/confirm', 'Uninstall', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 144, 222, 0, 0, 0, 0, 0, 0),
('admin', 223, 143, 'admin/build/menu/item/%/delete', 'admin/build/menu/item/%/delete', 'Delete menu item', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 143, 223, 0, 0, 0, 0, 0, 0),
('admin', 224, 143, 'admin/build/menu/item/%/edit', 'admin/build/menu/item/%/edit', 'Edit menu item', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 143, 224, 0, 0, 0, 0, 0, 0),
('admin', 225, 156, 'admin/content/taxonomy/edit/vocabulary/%', 'admin/content/taxonomy/edit/vocabulary/%', 'Edit vocabulary', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 119, 156, 225, 0, 0, 0, 0, 0, 0),
('admin', 226, 143, 'admin/build/menu/item/%/reset', 'admin/build/menu/item/%/reset', 'Reset menu item', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 143, 226, 0, 0, 0, 0, 0, 0),
('devel', 227, 0, 'admin/reports/status/run-cron', 'admin/reports/status/run-cron', 'Run cron', 'a:0:{}', 'devel', 0, 0, 0, 0, 0, 1, 0, 227, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('devel', 228, 0, 'admin/settings/devel', 'admin/settings', 'Devel settings', 'a:0:{}', 'devel', 0, 0, 0, 0, 0, 1, 0, 228, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 229, 0, 'documents', 'documents', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 229, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 230, 0, 'mimemail', 'mimemail', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 230, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 231, 0, 'imce', 'imce', 'File browser', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 231, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 232, 0, 'flag', 'flag', 'Flag', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 232, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 233, 0, 'rdf', 'rdf', 'RDF', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 233, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 234, 0, 'ckeditor-node-embed', 'ckeditor-node-embed', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 234, 0, 0, 0, 0, 0, 0, 0, 0, 0),
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('admin', 420, 0, 'admin/content/node-type/panel/delete', 'admin/content/node-type/panel/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 420, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 421, 0, 'admin/content/nodequeue/%/delete', 'admin/content/nodequeue/%/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 5, 1, 0, 421, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 422, 289, 'admin/build/custompage/delete/%', 'admin/build/custompage/delete/%', 'Delete Custom Page', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 289, 422, 0, 0, 0, 0, 0, 0),
('admin', 423, 296, 'admin/build/feeds/delete/%', 'admin/build/feeds/delete/%', 'Delete configuration', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 296, 423, 0, 0, 0, 0, 0, 0),
('admin', 424, 396, 'admin/settings/rdf/namespaces/delete', 'admin/settings/rdf/namespaces/delete', 'Delete namespace', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 5, 0, 114, 124, 318, 396, 424, 0, 0, 0, 0, 0),
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('admin', 430, 114, 'admin/views/ajax/autocomplete/tag', 'admin/views/ajax/autocomplete/tag', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 2, 0, 114, 430, 0, 0, 0, 0, 0, 0, 0, 0),
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('admin', 433, 135, 'admin/settings/date-time/delete/%', 'admin/settings/date-time/delete/%', 'Delete date format type', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:52:"Allow users to delete a configured date format type.";}}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 135, 433, 0, 0, 0, 0, 0, 0),
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('admin', 435, 351, 'admin/build/views/break-lock/%', 'admin/build/views/break-lock/%', 'Delete view', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 435, 0, 0, 0, 0, 0, 0),
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('admin', 437, 351, 'admin/build/views/clone/%', 'admin/build/views/clone/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 437, 0, 0, 0, 0, 0, 0),
('admin', 438, 351, 'admin/build/views/disable/%', 'admin/build/views/disable/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 438, 0, 0, 0, 0, 0, 0),
('admin', 439, 351, 'admin/build/views/enable/%', 'admin/build/views/enable/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 439, 0, 0, 0, 0, 0, 0),
('admin', 440, 310, 'admin/build/pages/edit/%', 'admin/build/pages/edit/%', 'Edit', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 310, 440, 0, 0, 0, 0, 0, 0),
('admin', 441, 330, 'admin/build/workflow/edit/%', 'admin/build/workflow/edit/%', 'Edit workflow', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 330, 441, 0, 0, 0, 0, 0, 0),
('admin', 442, 295, 'admin/build/features/export/populate', 'admin/build/features/export/populate', 'Populate feature', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:61:"AHAH callback to populate a feature from selected components.";}}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 295, 442, 0, 0, 0, 0, 0, 0),
('admin', 443, 0, 'admin/build/features/%/status', 'admin/build/features/%/status', 'Status', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:28:"Javascript status call back.";}}', 'system', -1, 0, 0, 0, 0, 1, 0, 443, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 444, 310, 'admin/build/pages/wizard/%', 'admin/build/pages/wizard/%', 'Wizard', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 310, 444, 0, 0, 0, 0, 0, 0),
('admin', 445, 288, 'admin/build/context/list/%', 'admin/build/context/list/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 288, 445, 0, 0, 0, 0, 0, 0),
('admin', 446, 0, 'admin/build/imagecache/%/%/delete', 'admin/build/imagecache/%/%/delete', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 446, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 447, 135, 'admin/settings/date-time/formats/delete/%', 'admin/settings/date-time/formats/delete/%', 'Delete date format', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:47:"Allow users to delete a configured date format.";}}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 135, 447, 0, 0, 0, 0, 0, 0),
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('admin', 449, 0, 'admin/build/imagecache/%/add/%', 'admin/build/imagecache/%/add/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 449, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 450, 286, 'admin/settings/calais/queue/enqueue/%', 'admin/settings/calais/queue/enqueue/%', 'Calais Queue Processing', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:66:"Submits all Content of the specified type to Calais for processing";}}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 124, 286, 450, 0, 0, 0, 0, 0, 0),
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('admin', 452, 385, 'admin/settings/rdf/feeds/edit/%', 'admin/settings/rdf/feeds/edit/%', 'Configure RDF feed', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 5, 0, 114, 124, 318, 385, 452, 0, 0, 0, 0, 0),
('admin', 453, 351, 'admin/build/views/tools/export/results', 'admin/build/views/tools/export/results', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 453, 0, 0, 0, 0, 0, 0),
('admin', 454, 0, 'admin/content/nodequeue/%/clear/%', 'admin/content/nodequeue/%/clear/%', 'Clear', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 454, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 455, 399, 'admin/settings/rdf/repositories/rdf/delete', 'admin/settings/rdf/repositories/rdf/delete', 'Delete local repository', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 5, 0, 114, 124, 318, 399, 455, 0, 0, 0, 0, 0),
('admin', 456, 310, 'admin/build/pages/%/operation/%', 'admin/build/pages/%/operation/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 310, 456, 0, 0, 0, 0, 0, 0),
('admin', 457, 351, 'admin/build/views/%/add-display/%', 'admin/build/views/%/add-display/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 457, 0, 0, 0, 0, 0, 0),
('admin', 458, 351, 'admin/build/views/%/%/%', 'admin/build/views/%/%/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 458, 0, 0, 0, 0, 0, 0),
('admin', 459, 351, 'admin/build/views/%/analyze/%', 'admin/build/views/%/analyze/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 459, 0, 0, 0, 0, 0, 0),
('admin', 460, 330, 'admin/build/workflow/state/disable/%', 'admin/build/workflow/state/disable/%', 'Disable State', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 330, 460, 0, 0, 0, 0, 0, 0),
('admin', 461, 310, 'admin/build/pages/%/enable/%', 'admin/build/pages/%/enable/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 310, 461, 0, 0, 0, 0, 0, 0),
('admin', 462, 351, 'admin/build/views/%/details/%', 'admin/build/views/%/details/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 462, 0, 0, 0, 0, 0, 0),
('admin', 463, 310, 'admin/build/pages/%/disable/%', 'admin/build/pages/%/disable/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 310, 463, 0, 0, 0, 0, 0, 0),
('admin', 464, 399, 'admin/settings/rdf/repositories/rdf/edit', 'admin/settings/rdf/repositories/rdf/edit', 'Edit local repository', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 5, 0, 114, 124, 318, 399, 464, 0, 0, 0, 0, 0),
('admin', 465, 351, 'admin/build/views/%/preview/%', 'admin/build/views/%/preview/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 351, 465, 0, 0, 0, 0, 0, 0),
('admin', 466, 0, 'admin/build/context/list/%/clone', 'admin/build/context/list/%/clone', 'Clone', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 466, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 467, 0, 'admin/build/context/list/%/delete', 'admin/build/context/list/%/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 467, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 468, 0, 'admin/build/context/list/%/disable', 'admin/build/context/list/%/disable', 'Disable', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 468, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 469, 0, 'admin/build/context/list/%/enable', 'admin/build/context/list/%/enable', 'Enable', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 469, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 471, 0, 'admin/build/context/list/%/revert', 'admin/build/context/list/%/revert', 'Revert', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 471, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 472, 0, 'admin/content/nodequeue/%/add/%/%', 'admin/content/nodequeue/%/add/%/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 472, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 473, 0, 'admin/content/nodequeue/%/remove-node/%/%', 'admin/content/nodequeue/%/remove-node/%/%', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 473, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 485, 14, 'node/add/author', 'node/add/author', 'Author', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:131:"Author can be associated with a Drupal user, but can also be a contributing author that does not have Drupal login to your website.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 485, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 486, 14, 'node/add/page', 'node/add/page', 'Page', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:296:"A <em>page</em>, similar in form to a <em>story</em>, is a simple method for creating and displaying information that rarely changes, such as an "About us" section of a website. By default, a <em>page</em> entry does not allow visitor comments and is not featured on the site''s initial home page.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 486, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 487, 119, 'admin/content/node-type/author', 'admin/content/node-type/author', 'Author', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 119, 487, 0, 0, 0, 0, 0, 0, 0),
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('admin', 489, 0, 'admin/content/node-type/page/delete', 'admin/content/node-type/page/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 489, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 490, 0, 'admin/content/node-type/author/delete', 'admin/content/node-type/author/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 490, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 491, 0, 'articles', 'articles', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 491, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 492, 0, 'blogs', 'blogs', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 492, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 493, 0, 'topics', 'topics', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 493, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 494, 0, 'events', 'events', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 494, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 495, 0, 'multimedia', 'multimedia', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 495, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 496, 0, 'resources', 'resources', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 496, 0, 0, 0, 0, 0, 0, 0, 0, 0),
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('admin', 504, 122, 'admin/reports/updates', 'admin/reports/updates', '', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 122, 504, 0, 0, 0, 0, 0, 0, 0),
('navigation', 505, 14, 'node/add/article', 'node/add/article', 'Article', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:73:"Articles can be posted to the site in category lists and on the homepage.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 505, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 506, 14, 'node/add/audio', 'node/add/audio', 'Audio', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:112:"Audio content can be entered by referencing an external source, or by uploading a local audio file into the CMS.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 506, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 507, 498, 'admin/rules/autocomplete', 'admin/rules/autocomplete', '', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 498, 507, 0, 0, 0, 0, 0, 0, 0),
('admin', 508, 119, 'admin/content/clearcache', 'admin/content/clearcache', 'Clear cache', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, -49, 3, 1, 114, 119, 508, 0, 0, 0, 0, 0, 0, 0),
('navigation', 509, 14, 'node/add/editors-choice', 'node/add/editors-choice', 'Editor''s Choice', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:109:"This type of content can be created for display as a block on the Homepage, right sidebar and other sections.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 509, 0, 0, 0, 0, 0, 0, 0, 0),
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('admin', 513, 124, 'admin/settings/openpublish', 'admin/settings/openpublish', 'OpenPublish', 'a:1:{s:5:"admin";b:1;}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 124, 513, 0, 0, 0, 0, 0, 0, 0),
('navigation', 514, 14, 'node/add/package', 'node/add/package', 'Package', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:155:"News packages are sets of topical information based around one relevant event or topic. Examples might include an election, or coverage of the war in Iraq.";}}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 514, 0, 0, 0, 0, 0, 0, 0, 0),
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('admin', 617, 0, 'admin/content/node-type/audio/groups/%/remove', 'admin/content/node-type/audio/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 617, 0, 0, 0, 0, 0, 0, 0, 0, 0);
INSERT INTO `menu_links` (`menu_name`, `mlid`, `plid`, `link_path`, `router_path`, `link_title`, `options`, `module`, `hidden`, `external`, `has_children`, `expanded`, `weight`, `depth`, `customized`, `p1`, `p2`, `p3`, `p4`, `p5`, `p6`, `p7`, `p8`, `p9`, `updated`) VALUES
('admin', 618, 0, 'admin/content/node-type/author/groups/%/remove', 'admin/content/node-type/author/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 618, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 619, 0, 'admin/content/node-type/blog/groups/%/remove', 'admin/content/node-type/blog/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 619, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 620, 0, 'admin/content/node-type/editors-choice/groups/%/remove', 'admin/content/node-type/editors-choice/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 620, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 621, 0, 'admin/content/node-type/event/groups/%/remove', 'admin/content/node-type/event/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 621, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 622, 0, 'admin/content/node-type/op-image/groups/%/remove', 'admin/content/node-type/op-image/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 622, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 623, 0, 'admin/content/node-type/package/groups/%/remove', 'admin/content/node-type/package/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 623, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 624, 0, 'admin/content/node-type/page/groups/%/remove', 'admin/content/node-type/page/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 624, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 625, 0, 'admin/content/node-type/panel/groups/%/remove', 'admin/content/node-type/panel/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 625, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 626, 0, 'admin/content/node-type/resource/groups/%/remove', 'admin/content/node-type/resource/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 626, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 627, 0, 'admin/content/node-type/slideshow/groups/%/remove', 'admin/content/node-type/slideshow/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 627, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 628, 0, 'admin/content/node-type/topichub/groups/%/remove', 'admin/content/node-type/topichub/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 628, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 629, 0, 'admin/content/node-type/video/groups/%/remove', 'admin/content/node-type/video/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 629, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 630, 0, 'admin/content/node-type/article/fields/field_deck/remove', 'admin/content/node-type/article/fields/field_deck/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 630, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 631, 0, 'admin/content/node-type/article/fields/field_main_image/remove', 'admin/content/node-type/article/fields/field_main_image/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 631, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 633, 0, 'admin/content/node-type/article/fields/field_op_author/remove', 'admin/content/node-type/article/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 633, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 635, 0, 'admin/content/node-type/article/fields/field_op_related_mmedia/remove', 'admin/content/node-type/article/fields/field_op_related_mmedia/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 635, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 636, 0, 'admin/content/node-type/article/fields/field_related_content/remove', 'admin/content/node-type/article/fields/field_related_content/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 636, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 640, 0, 'admin/content/node-type/audio/fields/field_audio_file/remove', 'admin/content/node-type/audio/fields/field_audio_file/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 640, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 641, 0, 'admin/content/node-type/audio/fields/field_embedded_audio/remove', 'admin/content/node-type/audio/fields/field_embedded_audio/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 641, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 642, 0, 'admin/content/node-type/audio/fields/field_op_audio_thumbnail/remove', 'admin/content/node-type/audio/fields/field_op_audio_thumbnail/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 642, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 643, 0, 'admin/content/node-type/audio/fields/field_op_author/remove', 'admin/content/node-type/audio/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 643, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 644, 0, 'admin/content/node-type/audio/fields/field_teaser/remove', 'admin/content/node-type/audio/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 644, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 645, 0, 'admin/content/node-type/author/fields/field_author_photo/remove', 'admin/content/node-type/author/fields/field_author_photo/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 645, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 646, 0, 'admin/content/node-type/author/fields/field_author_user/remove', 'admin/content/node-type/author/fields/field_author_user/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 646, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 647, 0, 'admin/content/node-type/author/fields/field_op_author_jobtitle/remove', 'admin/content/node-type/author/fields/field_op_author_jobtitle/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 647, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 648, 0, 'admin/content/node-type/blog/fields/field_op_author/remove', 'admin/content/node-type/blog/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 648, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 649, 0, 'admin/content/node-type/blog/fields/field_op_documentcloud_doc/remove', 'admin/content/node-type/blog/fields/field_op_documentcloud_doc/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 649, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 650, 0, 'admin/content/node-type/blog/fields/field_show_author_info/remove', 'admin/content/node-type/blog/fields/field_show_author_info/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 650, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 651, 0, 'admin/content/node-type/blog/fields/field_teaser/remove', 'admin/content/node-type/blog/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 651, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 652, 0, 'admin/content/node-type/editors-choice/fields/field_ec_featured_nodes/remove', 'admin/content/node-type/editors-choice/fields/field_ec_featured_nodes/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 652, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 653, 0, 'admin/content/node-type/event/fields/field_event_date/remove', 'admin/content/node-type/event/fields/field_event_date/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 653, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 654, 0, 'admin/content/node-type/event/fields/field_main_image/remove', 'admin/content/node-type/event/fields/field_main_image/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 654, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 655, 0, 'admin/content/node-type/event/fields/field_main_image_credit/remove', 'admin/content/node-type/event/fields/field_main_image_credit/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 655, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 656, 0, 'admin/content/node-type/event/fields/field_teaser/remove', 'admin/content/node-type/event/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 656, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 657, 0, 'admin/content/node-type/op-image/fields/field_main_image/remove', 'admin/content/node-type/op-image/fields/field_main_image/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 657, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 658, 0, 'admin/content/node-type/op-image/fields/field_op_embedded_image/remove', 'admin/content/node-type/op-image/fields/field_op_embedded_image/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 658, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 659, 0, 'admin/content/node-type/op-image/fields/field_op_image_credit/remove', 'admin/content/node-type/op-image/fields/field_op_image_credit/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 659, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 660, 0, 'admin/content/node-type/op-image/fields/field_teaser/remove', 'admin/content/node-type/op-image/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 660, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 661, 0, 'admin/content/node-type/package/fields/field_center_intro/remove', 'admin/content/node-type/package/fields/field_center_intro/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 661, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 662, 0, 'admin/content/node-type/package/fields/field_center_main_image/remove', 'admin/content/node-type/package/fields/field_center_main_image/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 662, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 663, 0, 'admin/content/node-type/package/fields/field_center_related/remove', 'admin/content/node-type/package/fields/field_center_related/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 663, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 664, 0, 'admin/content/node-type/package/fields/field_center_title/remove', 'admin/content/node-type/package/fields/field_center_title/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 664, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 665, 0, 'admin/content/node-type/package/fields/field_embedded_audio/remove', 'admin/content/node-type/package/fields/field_embedded_audio/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 665, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 666, 0, 'admin/content/node-type/package/fields/field_embedded_video/remove', 'admin/content/node-type/package/fields/field_embedded_video/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 666, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 667, 0, 'admin/content/node-type/package/fields/field_left_intro/remove', 'admin/content/node-type/package/fields/field_left_intro/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 667, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 668, 0, 'admin/content/node-type/package/fields/field_left_related/remove', 'admin/content/node-type/package/fields/field_left_related/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 668, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 669, 0, 'admin/content/node-type/package/fields/field_right_intro/remove', 'admin/content/node-type/package/fields/field_right_intro/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 669, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 670, 0, 'admin/content/node-type/package/fields/field_right_related/remove', 'admin/content/node-type/package/fields/field_right_related/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 670, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 671, 0, 'admin/content/node-type/resource/fields/field_links/remove', 'admin/content/node-type/resource/fields/field_links/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 671, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 672, 0, 'admin/content/node-type/resource/fields/field_op_author/remove', 'admin/content/node-type/resource/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 672, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 673, 0, 'admin/content/node-type/resource/fields/field_teaser/remove', 'admin/content/node-type/resource/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 673, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 674, 0, 'admin/content/node-type/slideshow/fields/field_op_author/remove', 'admin/content/node-type/slideshow/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 674, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 675, 0, 'admin/content/node-type/slideshow/fields/field_op_slideshow_images/remove', 'admin/content/node-type/slideshow/fields/field_op_slideshow_images/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 675, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 676, 0, 'admin/content/node-type/slideshow/fields/field_op_slideshow_thumb/remove', 'admin/content/node-type/slideshow/fields/field_op_slideshow_thumb/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 676, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 677, 0, 'admin/content/node-type/slideshow/fields/field_teaser/remove', 'admin/content/node-type/slideshow/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 677, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 678, 0, 'admin/content/node-type/video/fields/field_embedded_video/remove', 'admin/content/node-type/video/fields/field_embedded_video/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 678, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 679, 0, 'admin/content/node-type/video/fields/field_flash_file/remove', 'admin/content/node-type/video/fields/field_flash_file/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 679, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 680, 0, 'admin/content/node-type/video/fields/field_op_author/remove', 'admin/content/node-type/video/fields/field_op_author/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 680, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 681, 0, 'admin/content/node-type/video/fields/field_op_video_thumbnail/remove', 'admin/content/node-type/video/fields/field_op_video_thumbnail/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 681, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 682, 0, 'admin/content/node-type/video/fields/field_teaser/remove', 'admin/content/node-type/video/fields/field_teaser/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 682, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 683, 14, 'node/add/issue', 'node/add/issue', 'Issue', 'a:0:{}', 'system', 0, 0, 0, 0, 0, 2, 0, 14, 683, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 684, 119, 'admin/content/node-type/issue', 'admin/content/node-type/issue', 'Issue', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 119, 684, 0, 0, 0, 0, 0, 0, 0),
('admin', 685, 0, 'admin/content/node-type/issue/delete', 'admin/content/node-type/issue/delete', 'Delete', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 685, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 686, 0, 'admin/content/node-type/issue/groups/%', 'admin/content/node-type/issue/groups/%', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 686, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 687, 0, 'admin/content/node-type/issue/groups/%/remove', 'admin/content/node-type/issue/groups/%/remove', 'Edit group', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 687, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 688, 0, 'admin/content/node-type/issue/fields/field_date/remove', 'admin/content/node-type/issue/fields/field_date/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 688, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 689, 0, 'admin/content/node-type/issue/fields/field_issue_no/remove', 'admin/content/node-type/issue/fields/field_issue_no/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 689, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 690, 0, 'admin/content/node-type/issue/fields/field_volume/remove', 'admin/content/node-type/issue/fields/field_volume/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 690, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 697, 0, 'admin/content/node-type/article/fields/field_article_no/remove', 'admin/content/node-type/article/fields/field_article_no/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 697, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 698, 0, 'admin/content/node-type/article/fields/field_cor_add/remove', 'admin/content/node-type/article/fields/field_cor_add/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 698, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 699, 0, 'admin/content/node-type/article/fields/field_issue/remove', 'admin/content/node-type/article/fields/field_issue/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 699, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 700, 0, 'admin/content/node-type/article/fields/field_page/remove', 'admin/content/node-type/article/fields/field_page/remove', 'Remove field', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 1, 0, 700, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('navigation', 701, 0, 'contact', 'contact', 'Contact', 'a:0:{}', 'system', 1, 0, 0, 0, -46, 1, 1, 701, 0, 0, 0, 0, 0, 0, 0, 0, 0),
('admin', 702, 123, 'admin/build/contact', 'admin/build/contact', 'Contact form', 'a:1:{s:10:"attributes";a:1:{s:5:"title";s:71:"Create a system contact form and set up categories for the form to use.";}}', 'system', 0, 0, 0, 0, 0, 3, 0, 114, 123, 702, 0, 0, 0, 0, 0, 0, 0),
('admin', 703, 120, 'admin/help/contact', 'admin/help/contact', 'contact', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 3, 0, 114, 120, 703, 0, 0, 0, 0, 0, 0, 0),
('admin', 704, 297, 'admin/build/flags/actions/add', 'admin/build/flags/actions/add', 'Add action', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 297, 704, 0, 0, 0, 0, 0, 0),
('admin', 705, 297, 'admin/build/flags/actions/delete', 'admin/build/flags/actions/delete', 'Delete action', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 297, 705, 0, 0, 0, 0, 0, 0),
('admin', 706, 702, 'admin/build/contact/delete/%', 'admin/build/contact/delete/%', 'Delete contact', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 702, 706, 0, 0, 0, 0, 0, 0),
('admin', 707, 297, 'admin/build/flags/actions/configure', 'admin/build/flags/actions/configure', 'Edit action', 'a:0:{}', 'system', -1, 0, 0, 0, 0, 4, 0, 114, 123, 297, 707, 0, 0, 0, 0, 0, 0),
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-- --------------------------------------------------------

--
-- Table structure for table `menu_router`
--

CREATE TABLE IF NOT EXISTS `menu_router` (
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  `title` varchar(255) NOT NULL DEFAULT '',
  `title_callback` varchar(255) NOT NULL DEFAULT '',
  `title_arguments` varchar(255) NOT NULL DEFAULT '',
  `type` int(11) NOT NULL DEFAULT '0',
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--
-- Dumping data for table `menu_router`
--

INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
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INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
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('admin/build/menu/add', '', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'drupal_get_form', 'a:2:{i:0;s:14:"menu_edit_menu";i:1;s:3:"add";}', 15, 4, 'admin/build/menu', 'admin/build/menu', 'Add menu', 't', '', 128, '', '', '', 0, 'modules/menu/menu.admin.inc'),
('admin/build/menu/item/%/delete', 'a:1:{i:4;s:14:"menu_link_load";}', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'menu_item_delete_page', 'a:1:{i:0;i:4;}', 61, 6, '', 'admin/build/menu/item/%/delete', 'Delete menu item', 't', '', 4, '', '', '', 0, 'modules/menu/menu.admin.inc'),
('admin/build/menu/item/%/edit', 'a:1:{i:4;s:14:"menu_link_load";}', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'drupal_get_form', 'a:4:{i:0;s:14:"menu_edit_item";i:1;s:4:"edit";i:2;i:4;i:3;N;}', 61, 6, '', 'admin/build/menu/item/%/edit', 'Edit menu item', 't', '', 4, '', '', '', 0, 'modules/menu/menu.admin.inc'),
('admin/build/menu/item/%/reset', 'a:1:{i:4;s:14:"menu_link_load";}', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'drupal_get_form', 'a:2:{i:0;s:23:"menu_reset_item_confirm";i:1;i:4;}', 61, 6, '', 'admin/build/menu/item/%/reset', 'Reset menu item', 't', '', 4, '', '', '', 0, 'modules/menu/menu.admin.inc'),
('admin/build/menu/list', '', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'menu_overview_page', 'a:0:{}', 15, 4, 'admin/build/menu', 'admin/build/menu', 'List menus', 't', '', 136, '', '', '', -10, 'modules/menu/menu.admin.inc'),
('admin/build/menu/settings', '', '', 'user_access', 'a:1:{i:0;s:15:"administer menu";}', 'drupal_get_form', 'a:1:{i:0;s:14:"menu_configure";}', 15, 4, 'admin/build/menu', 'admin/build/menu', 'Settings', 't', '', 128, '', '', '', 5, 'modules/menu/menu.admin.inc'),
('admin/build/modules', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:14:"system_modules";}', 7, 3, '', 'admin/build/modules', 'Modules', 't', '', 6, '', 'Enable or disable add-on modules for your site.', '', 0, 'modules/system/system.admin.inc'),
('admin/build/modules/list', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:14:"system_modules";}', 15, 4, 'admin/build/modules', 'admin/build/modules', 'List', 't', '', 136, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/build/modules/list/confirm', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:14:"system_modules";}', 31, 5, '', 'admin/build/modules/list/confirm', 'List', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/build/modules/uninstall', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:24:"system_modules_uninstall";}', 15, 4, 'admin/build/modules', 'admin/build/modules', 'Uninstall', 't', '', 128, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/build/modules/uninstall/confirm', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:24:"system_modules_uninstall";}', 31, 5, '', 'admin/build/modules/uninstall/confirm', 'Uninstall', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/build/pages', '', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_list_page', 'a:0:{}', 7, 3, '', 'admin/build/pages', 'Pages', 't', '', 6, '', 'Add, edit and remove overridden system pages and user defined pages from the system.', '', 0, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/%/disable/%', 'a:2:{i:3;s:14:"ctools_js_load";i:5;s:23:"page_manager_cache_load";}', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_enable_page', 'a:3:{i:0;b:1;i:1;i:3;i:2;i:5;}', 58, 6, '', 'admin/build/pages/%/disable/%', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/%/enable/%', 'a:2:{i:3;s:14:"ctools_js_load";i:5;s:23:"page_manager_cache_load";}', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_enable_page', 'a:3:{i:0;b:0;i:1;i:3;i:2;i:5;}', 58, 6, '', 'admin/build/pages/%/enable/%', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/%/operation/%', 'a:2:{i:3;s:14:"ctools_js_load";i:5;s:23:"page_manager_cache_load";}', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_edit_page_operation', 'a:2:{i:0;i:3;i:1;i:5;}', 58, 6, '', 'admin/build/pages/%/operation/%', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/add', '', '', 'user_access', 'a:1:{i:0;s:23:"administer page manager";}', 'page_manager_page_add_subtask', 'a:0:{}', 15, 4, 'admin/build/pages', 'admin/build/pages', 'Add custom page', 't', '', 128, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/page.admin.inc'),
('admin/build/pages/argument', '', '', 'user_access', 'a:1:{i:0;s:23:"administer page manager";}', 'page_manager_page_subtask_argument_ajax', 'a:0:{}', 15, 4, '', 'admin/build/pages/argument', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/page.admin.inc'),
('admin/build/pages/edit/%', 'a:1:{i:4;s:23:"page_manager_cache_load";}', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_edit_page', 'a:1:{i:0;i:4;}', 30, 5, '', 'admin/build/pages/edit/%', 'Edit', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/import', '', '', 'ctools_access_multiperm', 'a:2:{i:0;s:23:"administer page manager";i:1;s:28:"use PHP for block visibility";}', 'drupal_get_form', 'a:2:{i:0;s:32:"page_manager_page_import_subtask";i:1;s:4:"page";}', 15, 4, 'admin/build/pages', 'admin/build/pages', 'Import page', 't', '', 128, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/page.admin.inc'),
('admin/build/pages/list', '', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_list_page', 'a:0:{}', 15, 4, 'admin/build/pages', 'admin/build/pages', 'List', 't', '', 136, '', '', '', -10, 'sites/all/modules/ctools/page_manager/page_manager.admin.inc'),
('admin/build/pages/wizard', '', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_page_wizard_list', 'a:1:{i:0;i:4;}', 15, 4, 'admin/build/pages', 'admin/build/pages', 'Wizards', 't', '', 128, '', '', '', -5, 'sites/all/modules/ctools/includes/page-wizard.inc'),
('admin/build/pages/wizard/%', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:16:"use page manager";}', 'page_manager_page_wizard', 'a:1:{i:0;i:4;}', 30, 5, '', 'admin/build/pages/wizard/%', 'Wizard', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/page-wizard.inc'),
('admin/build/panels', '', '', 'user_access', 'a:1:{i:0;s:20:"use panels dashboard";}', 'panels_admin_page', 'a:0:{}', 7, 3, '', 'admin/build/panels', 'Panels', 't', '', 6, '', 'Get a bird''s eye view of items related to Panels.', '', 0, 'sites/all/modules/panels/includes/callbacks.inc'),
('admin/build/panels/dashboard', '', '', 'user_access', 'a:1:{i:0;s:20:"use panels dashboard";}', 'panels_admin_page', 'a:0:{}', 15, 4, 'admin/build/panels', 'admin/build/panels', 'Dashboard', 't', '', 136, '', '', '', -10, 'sites/all/modules/panels/includes/callbacks.inc'),
('admin/build/panels/layouts', '', '', 'ctools_export_ui_task_access', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:4:"list";}', 'ctools_export_ui_switcher_page', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:4:"list";}', 15, 4, 'admin/build/panels', 'admin/build/panels', 'Layouts', 't', '', 128, '', 'Add, edit or delete custom content layouts.', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/add-flexible', '', '', 'ctools_export_ui_task_access', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:3:"add";}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:3:"add";i:2;s:8:"flexible";}', 31, 5, 'admin/build/panels/layouts', 'admin/build/panels', 'Add flexible layout', 't', '', 128, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/import', '', '', 'ctools_export_ui_task_access', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:6:"import";}', 'ctools_export_ui_switcher_page', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:6:"import";}', 31, 5, 'admin/build/panels/layouts', 'admin/build/panels', 'Import', 't', '', 128, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list', '', '', 'ctools_export_ui_task_access', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:4:"list";}', 'ctools_export_ui_switcher_page', 'a:2:{i:0;s:14:"panels_layouts";i:1;s:4:"list";}', 31, 5, 'admin/build/panels/layouts', 'admin/build/panels', 'List', 't', '', 136, '', '', '', -10, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:4:"edit";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:4:"edit";i:2;i:5;}', 62, 6, '', 'admin/build/panels/layouts/list/%', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/clone', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:5:"clone";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:5:"clone";i:2;i:5;}', 125, 7, '', 'admin/build/panels/layouts/list/%/clone', 'Clone', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/delete', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"delete";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"delete";i:2;i:5;}', 125, 7, '', 'admin/build/panels/layouts/list/%/delete', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/disable', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:7:"disable";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:7:"disable";i:2;i:5;}', 125, 7, '', 'admin/build/panels/layouts/list/%/disable', 'Disable', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/edit', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:4:"edit";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:4:"edit";i:2;i:5;}', 125, 7, 'admin/build/panels/layouts/list/%', 'admin/build/panels/layouts/list/%', 'Edit', 't', '', 136, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/enable', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"enable";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"enable";i:2;i:5;}', 125, 7, '', 'admin/build/panels/layouts/list/%/enable', 'Enable', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/export', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"export";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"export";i:2;i:5;}', 125, 7, 'admin/build/panels/layouts/list/%', 'admin/build/panels/layouts/list/%', 'Export', 't', '', 128, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/layouts/list/%/revert', 'a:1:{i:5;a:1:{s:21:"ctools_export_ui_load";a:1:{i:0;s:14:"panels_layouts";}}}', '', 'ctools_export_ui_task_access', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"revert";i:2;i:5;}', 'ctools_export_ui_switcher_page', 'a:3:{i:0;s:14:"panels_layouts";i:1;s:6:"delete";i:2;i:5;}', 125, 7, '', 'admin/build/panels/layouts/list/%/revert', 'Revert', 't', '', 4, '', '', '', 0, 'sites/all/modules/ctools/includes/export-ui.inc'),
('admin/build/panels/settings', '', '', 'user_access', 'a:1:{i:0;s:20:"use panels dashboard";}', 'drupal_get_form', 'a:1:{i:0;s:26:"panels_admin_settings_page";}', 15, 4, 'admin/build/panels', 'admin/build/panels', 'Settings', 't', '', 128, '', '', '', 0, 'sites/all/modules/panels/includes/callbacks.inc'),
('admin/build/panels/settings/general', '', '', 'user_access', 'a:1:{i:0;s:23:"administer page manager";}', 'drupal_get_form', 'a:1:{i:0;s:26:"panels_admin_settings_page";}', 31, 5, 'admin/build/panels/settings', 'admin/build/panels', 'General', 't', '', 136, '', '', '', -10, 'sites/all/modules/panels/includes/callbacks.inc'),
('admin/build/panels/settings/panel-node', '', '', 'user_access', 'a:1:{i:0;s:22:"administer panel-nodes";}', 'panels_node_settings', 'a:0:{}', 31, 5, 'admin/build/panels/settings', 'admin/build/panels', 'Panel nodes', 't', '', 128, '', 'Configure which content is available to add to panel node displays.', '', 0, ''),
('admin/build/panels/settings/panel-page', '', '', 'user_access', 'a:1:{i:0;s:20:"use panels dashboard";}', 'panels_admin_panel_context_page', 'a:0:{}', 31, 5, 'admin/build/panels/settings', 'admin/build/panels', 'Panel pages', 't', '', 128, '', '', '', -10, 'sites/all/modules/panels/includes/callbacks.inc'),
('admin/build/path', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'path_admin_overview', 'a:0:{}', 7, 3, '', 'admin/build/path', 'URL aliases', 't', '', 6, '', 'Change your site''s URL paths by aliasing them.', '', 0, 'modules/path/path.admin.inc'),
('admin/build/path/add', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'path_admin_edit', 'a:0:{}', 15, 4, 'admin/build/path', 'admin/build/path', 'Add alias', 't', '', 128, '', '', '', 0, 'modules/path/path.admin.inc'),
('admin/build/path/delete', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'drupal_get_form', 'a:1:{i:0;s:25:"path_admin_delete_confirm";}', 15, 4, '', 'admin/build/path/delete', 'Delete alias', 't', '', 4, '', '', '', 0, 'modules/path/path.admin.inc'),
('admin/build/path/delete_bulk', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'drupal_get_form', 'a:1:{i:0;s:21:"pathauto_admin_delete";}', 15, 4, 'admin/build/path', 'admin/build/path', 'Delete aliases', 't', '', 128, '', '', '', 30, 'sites/all/modules/pathauto/pathauto.admin.inc'),
('admin/build/path/edit', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'path_admin_edit', 'a:0:{}', 15, 4, '', 'admin/build/path/edit', 'Edit alias', 't', '', 4, '', '', '', 0, 'modules/path/path.admin.inc'),
('admin/build/path/list', '', '', 'user_access', 'a:1:{i:0;s:22:"administer url aliases";}', 'path_admin_overview', 'a:0:{}', 15, 4, 'admin/build/path', 'admin/build/path', 'List', 't', '', 136, '', '', '', -10, 'modules/path/path.admin.inc'),
('admin/build/path/pathauto', '', '', 'user_access', 'a:1:{i:0;s:19:"administer pathauto";}', 'drupal_get_form', 'a:1:{i:0;s:23:"pathauto_admin_settings";}', 15, 4, 'admin/build/path', 'admin/build/path', 'Automated alias settings', 't', '', 128, '', '', '', 10, 'sites/all/modules/pathauto/pathauto.admin.inc'),
('admin/build/themes', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:18:"system_themes_form";i:1;N;}', 7, 3, '', 'admin/build/themes', 'Themes', 't', '', 6, '', 'Change which theme your site uses or allows users to set.', '', 0, 'modules/system/system.admin.inc'),
('admin/build/themes/select', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:18:"system_themes_form";i:1;N;}', 15, 4, 'admin/build/themes', 'admin/build/themes', 'List', 't', '', 136, '', 'Select the default theme.', '', -1, 'modules/system/system.admin.inc'),
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('admin/content/node-type/audio/fields/field_embedded_audio', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"audio";i:2;s:20:"field_embedded_audio";}', 63, 6, 'admin/content/node-type/audio/fields', 'admin/content/node-type/audio', 'Embedded Audio', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
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('admin/content/node-type/audio/fields/field_op_audio_thumbnail', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"audio";i:2;s:24:"field_op_audio_thumbnail";}', 63, 6, 'admin/content/node-type/audio/fields', 'admin/content/node-type/audio', 'Audio Thumbnail', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
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('admin/content/node-type/audio/fields/field_op_author', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"audio";i:2;s:15:"field_op_author";}', 63, 6, 'admin/content/node-type/audio/fields', 'admin/content/node-type/audio', 'Author(s)', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc');
INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
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('admin/content/node-type/audio/groups/%/remove', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"fieldgroup_remove_group";i:1;s:5:"audio";i:2;i:5;}', 125, 7, '', 'admin/content/node-type/audio/groups/%/remove', 'Edit group', 't', '', 4, '', '', '', 0, ''),
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('admin/content/node-type/issue/fields/field_volume', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"issue";i:2;s:12:"field_volume";}', 63, 6, 'admin/content/node-type/issue/fields', 'admin/content/node-type/issue', 'Volume No', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
('admin/content/node-type/issue/fields/field_volume/remove', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:25:"content_field_remove_form";i:1;s:5:"issue";i:2;s:12:"field_volume";}', 127, 7, '', 'admin/content/node-type/issue/fields/field_volume/remove', 'Remove field', 't', '', 4, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
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('admin/content/node-type/video/display/token', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:29:"content_display_overview_form";i:1;s:5:"video";i:2;s:5:"token";}', 63, 6, 'admin/content/node-type/video/display', 'admin/content/node-type/video', 'Token', 't', '', 128, '', '', '', 1, 'sites/all/modules/cck/includes/content.admin.inc');
INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
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('admin/content/node-type/video/fields/field_flash_file', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"video";i:2;s:16:"field_flash_file";}', 63, 6, 'admin/content/node-type/video/fields', 'admin/content/node-type/video', 'Flash File', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
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('admin/content/node-type/video/fields/field_op_video_thumbnail/remove', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:25:"content_field_remove_form";i:1;s:5:"video";i:2;s:24:"field_op_video_thumbnail";}', 127, 7, '', 'admin/content/node-type/video/fields/field_op_video_thumbnail/remove', 'Remove field', 't', '', 4, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
('admin/content/node-type/video/fields/field_teaser', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"content_field_edit_form";i:1;s:5:"video";i:2;s:12:"field_teaser";}', 63, 6, 'admin/content/node-type/video/fields', 'admin/content/node-type/video', 'Teaser', 't', '', 128, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
('admin/content/node-type/video/fields/field_teaser/remove', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:25:"content_field_remove_form";i:1;s:5:"video";i:2;s:12:"field_teaser";}', 127, 7, '', 'admin/content/node-type/video/fields/field_teaser/remove', 'Remove field', 't', '', 4, '', '', '', 0, 'sites/all/modules/cck/includes/content.admin.inc'),
('admin/content/node-type/video/groups/%', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:26:"fieldgroup_group_edit_form";i:1;s:5:"video";i:2;i:5;}', 62, 6, '', 'admin/content/node-type/video/groups/%', 'Edit group', 't', '', 4, '', '', '', 0, ''),
('admin/content/node-type/video/groups/%/remove', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:3:{i:0;s:23:"fieldgroup_remove_group";i:1;s:5:"video";i:2;i:5;}', 125, 7, '', 'admin/content/node-type/video/groups/%/remove', 'Edit group', 't', '', 4, '', '', '', 0, ''),
('admin/content/node-type/video/relationships', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'noderelationships_admin_page', 'a:2:{i:0;i:5;i:1;s:5:"video";}', 31, 5, 'admin/content/node-type/video', 'admin/content/node-type/video', 'Relationships', 't', '', 128, '', '', '', 10, 'sites/all/modules/noderelationships/noderelationships.admin.inc'),
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('admin/content/node-type/video/relationships/erd', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'noderelationships_admin_page', 'a:2:{i:0;i:5;i:1;s:5:"video";}', 63, 6, 'admin/content/node-type/video/relationships', 'admin/content/node-type/video', 'Entity relations diagram', 't', '', 136, '', '', '', 0, 'sites/all/modules/noderelationships/noderelationships.admin.inc'),
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('admin/content/nodequeue/%/delete', 'a:1:{i:3;s:14:"nodequeue_load";}', '', 'user_access', 'a:1:{i:0;s:20:"administer nodequeue";}', 'drupal_get_form', 'a:2:{i:0;s:22:"nodequeue_admin_delete";i:1;i:3;}', 29, 5, '', 'admin/content/nodequeue/%/delete', 'Delete', 't', '', 4, '', '', '', 5, ''),
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('admin/content/nodequeue/%/remove-node/%/%', 'a:3:{i:3;s:14:"nodequeue_load";i:5;s:13:"subqueue_load";i:6;s:9:"node_load";}', '', 'nodequeue_node_and_queue_access', 'a:3:{i:0;i:6;i:1;i:3;i:2;i:5;}', 'nodequeue_admin_remove_node', 'a:3:{i:0;i:3;i:1;i:5;i:2;i:6;}', 116, 7, '', 'admin/content/nodequeue/%/remove-node/%/%', '', 't', '', 4, '', '', '', 0, ''),
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('admin/content/nodequeue/add/nodequeue', '', '', 'user_access', 'a:1:{i:0;s:20:"administer nodequeue";}', 'drupal_get_form', 'a:2:{i:0;s:25:"nodequeue_edit_queue_form";i:1;s:9:"nodequeue";}', 31, 5, 'admin/content/nodequeue', 'admin/content/nodequeue', 'Add @type', 't', 'a:1:{s:5:"@type";s:9:"nodequeue";}', 128, '', '', '', 0, ''),
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('admin/content/nodequeue/settings', '', '', 'user_access', 'a:1:{i:0;s:20:"administer nodequeue";}', 'drupal_get_form', 'a:1:{i:0;s:24:"nodequeue_admin_settings";}', 15, 4, 'admin/content/nodequeue', 'admin/content/nodequeue', 'Settings', 't', '', 128, '', '', '', 0, ''),
('admin/content/node_import', '', '', 'user_access', 'a:1:{i:0;s:14:"import content";}', 'drupal_get_form', 'a:1:{i:0;s:27:"node_import_list_tasks_form";}', 7, 3, '', 'admin/content/node_import', 'Import content', 't', '', 6, '', 'Import content from a <abbr title="Comma Separated Values">CSV</abbr> or <abbr title="Tab Separated Values">TSV</abbr> file.', '', 0, 'sites/all/modules/node_import/node_import.admin.inc'),
('admin/content/node_import/%', 'a:1:{i:3;s:16:"node_import_load";}', '', 'node_import_access', 'a:1:{i:0;i:3;}', 'drupal_get_form', 'a:2:{i:0;s:21:"node_import_view_form";i:1;i:3;}', 14, 4, '', 'admin/content/node_import/%', 'Importing...', 't', '', 4, '', '', '', 0, 'sites/all/modules/node_import/node_import.admin.inc'),
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('admin/content/node_import/add', '', '', 'user_access', 'a:1:{i:0;s:14:"import content";}', 'drupal_get_form', 'a:1:{i:0;s:20:"node_import_add_form";}', 15, 4, 'admin/content/node_import', 'admin/content/node_import', 'New import', 't', '', 128, '', '', '', 0, 'sites/all/modules/node_import/node_import.admin.inc'),
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('admin/content/node_import/list/files', '', '', 'user_access', 'a:1:{i:0;s:14:"import content";}', 'drupal_get_form', 'a:1:{i:0;s:27:"node_import_list_files_form";}', 31, 5, 'admin/content/node_import/list', 'admin/content/node_import', 'Files', 't', '', 128, '', '', '', 0, 'sites/all/modules/node_import/node_import.admin.inc'),
('admin/content/node_import/list/tasks', '', '', 'user_access', 'a:1:{i:0;s:14:"import content";}', 'drupal_get_form', 'a:1:{i:0;s:27:"node_import_list_tasks_form";}', 31, 5, 'admin/content/node_import/list', 'admin/content/node_import', 'Tasks', 't', '', 136, '', '', '', -10, 'sites/all/modules/node_import/node_import.admin.inc'),
('admin/content/node_import/settings', '', '', 'user_access', 'a:1:{i:0;s:18:"administer imports";}', 'drupal_get_form', 'a:1:{i:0;s:25:"node_import_settings_form";}', 15, 4, 'admin/content/node_import', 'admin/content/node_import', 'Settings', 't', '', 128, '', '', '', 10, 'sites/all/modules/node_import/node_import.admin.inc'),
('admin/content/rdf', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'drupal_get_form', 'a:1:{i:0;s:14:"rdf_admin_data";}', 7, 3, '', 'admin/content/rdf', 'RDF data', 't', '', 6, '', 'Query, create, and delete RDF statements.', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/content/rdf/add', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'drupal_get_form', 'a:1:{i:0;s:22:"rdf_db_admin_data_form";}', 15, 4, 'admin/content/rdf', 'admin/content/rdf', 'Add statement', 't', '', 128, '', '', '', 1, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/content/rdf/export', '', '', 'user_access', 'a:1:{i:0;s:15:"export RDF data";}', 'drupal_get_form', 'a:1:{i:0;s:23:"rdf_export_admin_screen";}', 15, 4, 'admin/content/rdf', 'admin/content/rdf', 'Export data', 't', '', 128, '', '', '', 30, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/content/rdf/import', '', '', 'user_access', 'a:1:{i:0;s:15:"import RDF data";}', 'drupal_get_form', 'a:1:{i:0;s:23:"rdf_import_admin_screen";}', 15, 4, 'admin/content/rdf', 'admin/content/rdf', 'Import data', 't', '', 128, '', '', '', 20, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/content/rdf/list', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'drupal_get_form', 'a:1:{i:0;s:14:"rdf_admin_data";}', 15, 4, 'admin/content/rdf', 'admin/content/rdf', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/content/rss-publishing', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"system_rss_feeds_settings";}', 7, 3, '', 'admin/content/rss-publishing', 'RSS publishing', 't', '', 6, '', 'Configure the number of items per feed and whether feeds should be titles/teasers/full-text.', '', 0, 'modules/system/system.admin.inc'),
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('admin/content/taxonomy/%', 'a:1:{i:3;s:24:"taxonomy_vocabulary_load";}', '', 'user_access', 'a:1:{i:0;s:19:"administer taxonomy";}', 'drupal_get_form', 'a:2:{i:0;s:23:"taxonomy_overview_terms";i:1;i:3;}', 14, 4, '', 'admin/content/taxonomy/%', 'List terms', 't', '', 4, '', '', '', 0, 'modules/taxonomy/taxonomy.admin.inc'),
('admin/content/taxonomy/%/add/term', 'a:1:{i:3;s:24:"taxonomy_vocabulary_load";}', '', 'user_access', 'a:1:{i:0;s:19:"administer taxonomy";}', 'taxonomy_add_term_page', 'a:1:{i:0;i:3;}', 59, 6, 'admin/content/taxonomy/%', 'admin/content/taxonomy/%', 'Add term', 't', '', 128, '', '', '', 0, 'modules/taxonomy/taxonomy.admin.inc'),
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('admin/journal/manage/featured_articles', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:3:{i:3;i:3;i:4;i:4;i:8;i:8;}}}}', 'views_page', 'a:2:{i:0;s:17:"featured_articles";i:1;s:6:"page_2";}', 15, 4, 'admin/journal/manage', 'admin/journal/manage', 'Featured Articles', 't', '', 128, '', '', '', 1, ''),
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('admin/reports', '', '', 'user_access', 'a:1:{i:0;s:19:"access site reports";}', 'system_admin_menu_block_page', 'a:0:{}', 3, 2, '', 'admin/reports', 'Reports', 't', '', 6, '', 'View reports from system logs and other status information.', 'left', 5, 'modules/system/system.admin.inc'),
('admin/reports/access/%', 'a:1:{i:3;N;}', '', 'user_access', 'a:1:{i:0;s:17:"access statistics";}', 'statistics_access_log', 'a:1:{i:0;i:3;}', 14, 4, '', 'admin/reports/access/%', 'Details', 't', '', 4, '', 'View access log.', '', 0, 'modules/statistics/statistics.admin.inc'),
('admin/reports/hits', '', '', 'user_access', 'a:1:{i:0;s:17:"access statistics";}', 'statistics_recent_hits', 'a:0:{}', 7, 3, '', 'admin/reports/hits', 'Recent hits', 't', '', 6, '', 'View pages that have recently been visited.', '', 0, 'modules/statistics/statistics.admin.inc'),
('admin/reports/mollom', '', '', '_mollom_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:19:"mollom_reports_page";}', 7, 3, '', 'admin/reports/mollom', 'Mollom statistics', 't', '', 6, '', 'Reports and usage statistics for the Mollom module.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/reports/pages', '', '', 'user_access', 'a:1:{i:0;s:17:"access statistics";}', 'statistics_top_pages', 'a:0:{}', 7, 3, '', 'admin/reports/pages', 'Top pages', 't', '', 6, '', 'View pages that have been hit frequently.', '', 1, 'modules/statistics/statistics.admin.inc'),
('admin/reports/referrers', '', '', 'user_access', 'a:1:{i:0;s:17:"access statistics";}', 'statistics_top_referrers', 'a:0:{}', 7, 3, '', 'admin/reports/referrers', 'Top referrers', 't', '', 6, '', 'View top referrers.', '', 0, 'modules/statistics/statistics.admin.inc'),
('admin/reports/search', '', '', 'user_access', 'a:1:{i:0;s:19:"access site reports";}', 'dblog_top', 'a:1:{i:0;s:6:"search";}', 7, 3, '', 'admin/reports/search', 'Top search phrases', 't', '', 6, '', 'View most popular search phrases.', '', 0, 'modules/dblog/dblog.admin.inc'),
('admin/reports/settings', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:34:"statistics_access_logging_settings";}', 7, 3, '', 'admin/reports/settings', 'Access log settings', 't', '', 6, '', 'Control details about what and how your site logs.', '', 3, 'modules/statistics/statistics.admin.inc'),
('admin/reports/status', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_status', 'a:0:{}', 7, 3, '', 'admin/reports/status', 'Status report', 't', '', 6, '', 'Get a status report about your site''s operation and any detected problems.', '', 10, 'modules/system/system.admin.inc'),
('admin/reports/status/php', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_php', 'a:0:{}', 15, 4, '', 'admin/reports/status/php', 'PHP', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/reports/status/run-cron', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_run_cron', 'a:0:{}', 15, 4, '', 'admin/reports/status/run-cron', 'Run cron', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/reports/status/sql', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_sql', 'a:0:{}', 15, 4, '', 'admin/reports/status/sql', 'SQL', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/reports/swftools', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'swftools_status', 'a:0:{}', 7, 3, '', 'admin/reports/swftools', 'SWF Tools status', 't', '', 6, '', 'Get an overview of the status of the SWF Tools module and its supporting files.', '', 9, 'sites/all/modules/swftools/swftools.admin.status.inc'),
('admin/reports/updates', '', '', '0', 'a:0:{}', 'system_admin_menu_block_page', 'a:0:{}', 7, 3, '', 'admin/reports/updates', '', 't', '', 6, '', '', '', 0, '/system.admin.inc'),
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('admin/rules/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'rules_admin_categories_autocomplete', 'a:0:{}', 7, 3, '', 'admin/rules/autocomplete', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/forms', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:24:"rules_forms_admin_events";}', 7, 3, '', 'admin/rules/forms', 'Form events', 't', '', 6, '', 'Configure Rules forms events.', '', 6, 'sites/all/modules/rules/rules_forms/rules_forms.admin.inc'),
('admin/rules/forms/%/activate', 'a:1:{i:3;N;}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'rules_forms_activate', 'a:1:{i:0;i:3;}', 29, 5, '', 'admin/rules/forms/%/activate', 'Activate events for a form', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_forms/rules_forms.admin.inc'),
('admin/rules/ie', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:23:"rules_admin_form_export";}', 7, 3, '', 'admin/rules/ie', 'Import / Export', 't', '', 6, '', 'Export your rules as text or import rules.', '', 5, 'sites/all/modules/rules/rules_admin/rules_admin.export.inc'),
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('admin/rules/ie/import', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:23:"rules_admin_form_import";}', 15, 4, 'admin/rules/ie', 'admin/rules/ie', 'Import', 't', '', 128, '', '', '', 7, 'sites/all/modules/rules/rules_admin/rules_admin.export.inc'),
('admin/rules/rules/%', 'a:1:{i:3;s:9:"rule_load";}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:26:"rules_admin_form_edit_rule";i:1;i:3;}', 14, 4, '', 'admin/rules/rules/%', '', 'rules_admin_rule_title', 'a:2:{i:0;s:19:"Editing rule @label";i:1;i:3;}', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rules/%/add', 'a:1:{i:3;s:9:"rule_load";}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:20:"rules_admin_form_add";i:1;i:3;}', 29, 5, '', 'admin/rules/rules/%/add', 'Add', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rules/%/clone', 'a:1:{i:3;s:9:"rule_load";}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:22:"rules_admin_form_clone";i:1;i:3;}', 29, 5, '', 'admin/rules/rules/%/clone', 'Clone rule', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rules/%/delete', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:28:"rules_admin_item_form_delete";i:1;i:2;i:2;i:3;}', 29, 5, '', 'admin/rules/rules/%/delete', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rules/%/delete/%', 'a:2:{i:3;a:1:{s:9:"rule_load";a:1:{i:0;i:3;}}i:5;a:1:{s:17:"rule_element_load";a:1:{i:0;i:3;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:23:"rules_admin_form_delete";i:1;i:3;i:2;i:5;}', 58, 6, '', 'admin/rules/rules/%/delete/%', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rules/%/edit/%', 'a:2:{i:3;a:1:{s:9:"rule_load";a:1:{i:0;i:3;}}i:5;a:1:{s:17:"rule_element_load";a:1:{i:0;i:3;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:21:"rules_admin_form_edit";i:1;i:3;i:2;i:5;}', 58, 6, '', 'admin/rules/rules/%/edit/%', 'Edit', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rules/%/revert', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:28:"rules_admin_item_form_revert";i:1;i:2;i:2;i:3;}', 29, 5, '', 'admin/rules/rules/%/revert', 'Revert', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'rules_admin_sets_overview', 'a:0:{}', 7, 3, '', 'admin/rules/rule_sets', 'Rule sets', 't', '', 6, '', 'Create and manage rule sets.', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc');
INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
('admin/rules/rule_sets/%', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:30:"rules_admin_form_edit_rule_set";i:1;i:3;}', 14, 4, '', 'admin/rules/rule_sets/%', '', 'rules_admin_item_title', 'a:1:{i:0;i:3;}', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/%/add', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:25:"rules_admin_form_add_rule";i:1;i:3;}', 29, 5, 'admin/rules/rule_sets/%', 'admin/rules/rule_sets/%', 'Add a new rule', 't', '', 128, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/rule_sets/%/delete', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:28:"rules_admin_item_form_delete";i:1;i:2;i:2;i:3;}', 29, 5, '', 'admin/rules/rule_sets/%/delete', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/%/list', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:30:"rules_admin_form_edit_rule_set";i:1;i:3;}', 29, 5, 'admin/rules/rule_sets/%', 'admin/rules/rule_sets/%', 'Overview', 't', '', 136, '', '', '', -5, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/%/revert', 'a:1:{i:3;a:1:{s:15:"rules_item_load";a:1:{i:0;i:2;}}}', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:3:{i:0;s:28:"rules_admin_item_form_revert";i:1;i:2;i:2;i:3;}', 29, 5, '', 'admin/rules/rule_sets/%/revert', 'Revert', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/add', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:29:"rules_admin_form_add_rule_set";i:1;i:2;}', 15, 4, 'admin/rules/rule_sets', 'admin/rules/rule_sets', 'Add a new rule set', 't', '', 128, '', '', '', 3, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/js', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'rules_admin_form_js', 'a:1:{i:0;s:43:"rules_admin_form_add_rule_set_add_arguments";}', 15, 4, '', 'admin/rules/rule_sets/js', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/rule_sets/list', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'rules_admin_sets_overview', 'a:0:{}', 15, 4, 'admin/rules/rule_sets', 'admin/rules/rule_sets', 'Overview', 't', '', 136, '', '', '', -5, 'sites/all/modules/rules/rules_admin/rules_admin.sets.inc'),
('admin/rules/settings', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:20:"rules_admin_settings";}', 7, 3, '', 'admin/rules/settings', 'Settings', 't', '', 6, '', 'Set display options, show/hide Rules messages.', '', 7, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/trigger', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:25:"rules_admin_form_overview";}', 7, 3, '', 'admin/rules/trigger', 'Triggered rules', 't', '', 6, '', 'Customize your site by configuring rules that are evaluated on events.', '', -2, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/trigger/add', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:2:{i:0;s:25:"rules_admin_form_add_rule";i:1;b:0;}', 15, 4, 'admin/rules/trigger', 'admin/rules/trigger', 'Add a new rule', 't', '', 128, '', '', '', 0, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/rules/trigger/list', '', '', 'user_access', 'a:1:{i:0;s:16:"administer rules";}', 'drupal_get_form', 'a:1:{i:0;s:25:"rules_admin_form_overview";}', 15, 4, 'admin/rules/trigger', 'admin/rules/trigger', 'Overview', 't', '', 136, '', '', '', -5, 'sites/all/modules/rules/rules_admin/rules_admin.rule_forms.inc'),
('admin/search/paper', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:3:{i:3;i:3;i:4;i:4;i:8;i:8;}}}}', 'views_page', 'a:2:{i:0;s:13:"author_center";i:1;s:6:"page_1";}', 7, 3, '', 'admin/search/paper', 'Search manuscript', 't', '', 6, '', '', '', 0, ''),
('admin/search/paper/author', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:3:{i:3;i:3;i:4;i:4;i:8;i:8;}}}}', 'views_page', 'a:2:{i:0;s:13:"author_center";i:1;s:6:"page_2";}', 15, 4, 'admin/search/paper', 'admin/search/paper', 'By Author(s)', 't', '', 128, '', 'Search paper by author(s)', '', 1, ''),
('admin/search/paper/id', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:3:{i:3;i:3;i:4;i:4;i:8;i:8;}}}}', 'views_page', 'a:2:{i:0;s:13:"author_center";i:1;s:6:"page_1";}', 15, 4, 'admin/search/paper', 'admin/search/paper', 'Manuscript ID', 't', '', 136, '', 'Search paper by manuscript ID', '', 0, ''),
('admin/settings', '', '', 'admin_landing_page_access', 'a:1:{i:0;s:14:"admin/settings";}', 'system_settings_overview', 'a:0:{}', 3, 2, '', 'admin/settings', 'Site configuration', 't', '', 6, '', 'Adjust basic site configuration options.', 'right', -5, 'modules/system/system.admin.inc'),
('admin/settings/accordion_menu', '', '', 'user_access', 'a:1:{i:0;s:25:"administer accordion menu";}', 'drupal_get_form', 'a:2:{i:0;s:28:"accordion_menu_settings_form";i:1;i:2;}', 7, 3, '', 'admin/settings/accordion_menu', 'Accordion Menu', 't', '', 6, '', 'Add and modify accordion menu blocks.', '', 0, 'sites/all/modules/accordion_menu/includes/setting.inc'),
('admin/settings/actions', '', '', 'user_access', 'a:1:{i:0;s:18:"administer actions";}', 'system_actions_manage', 'a:0:{}', 7, 3, '', 'admin/settings/actions', 'Actions', 't', '', 6, '', 'Manage the actions defined for your site.', '', 0, ''),
('admin/settings/actions/configure', '', '', 'user_access', 'a:1:{i:0;s:18:"administer actions";}', 'drupal_get_form', 'a:1:{i:0;s:24:"system_actions_configure";}', 15, 4, '', 'admin/settings/actions/configure', 'Configure an advanced action', 't', '', 4, '', '', '', 0, ''),
('admin/settings/actions/delete/%', 'a:1:{i:4;s:12:"actions_load";}', '', 'user_access', 'a:1:{i:0;s:18:"administer actions";}', 'drupal_get_form', 'a:2:{i:0;s:26:"system_actions_delete_form";i:1;i:4;}', 30, 5, '', 'admin/settings/actions/delete/%', 'Delete action', 't', '', 4, '', 'Delete an action.', '', 0, ''),
('admin/settings/actions/manage', '', '', 'user_access', 'a:1:{i:0;s:18:"administer actions";}', 'system_actions_manage', 'a:0:{}', 15, 4, 'admin/settings/actions', 'admin/settings/actions', 'Manage actions', 't', '', 136, '', 'Manage the actions defined for your site.', '', -2, ''),
('admin/settings/actions/orphan', '', '', 'user_access', 'a:1:{i:0;s:18:"administer actions";}', 'system_actions_remove_orphans', 'a:0:{}', 15, 4, '', 'admin/settings/actions/orphan', 'Remove orphans', 't', '', 4, '', '', '', 0, ''),
('admin/settings/admin', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:19:"admin_settings_form";}', 7, 3, '', 'admin/settings/admin', 'Administration tools', 't', '', 6, '', 'Settings for site administration tools.', '', 0, 'sites/all/modules/admin/admin.admin.inc'),
('admin/settings/admin/rebuild', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:22:"admin_settings_rebuild";}', 15, 4, 'admin/settings/admin', 'admin/settings/admin', 'Rebuild', 't', '', 128, '', 'Wipe and rebuild the administrative menu.', '', 10, 'sites/all/modules/admin/admin.admin.inc'),
('admin/settings/admin/settings', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:19:"admin_settings_form";}', 15, 4, 'admin/settings/admin', 'admin/settings/admin', 'Settings', 't', '', 136, '', 'Settings for site administration tools.', '', 0, 'sites/all/modules/admin/admin.admin.inc'),
('admin/settings/admin/theme', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:27:"system_admin_theme_settings";}', 15, 4, 'admin/settings/admin', 'admin/settings/admin', 'Administration theme', 't', '', 128, 'system_admin_theme_settings', 'Settings for how your administrative pages should look.', 'left', 10, 'modules/system/system.admin.inc'),
('admin/settings/advuser', '', '', 'user_access', 'a:1:{i:0;s:18:"administer advuser";}', 'drupal_get_form', 'a:1:{i:0;s:16:"advuser_settings";}', 7, 3, '', 'admin/settings/advuser', 'Advanced User', 't', '', 6, '', 'Advanced User Settings', '', 0, 'sites/all/modules/advuser/advuser_filters.inc'),
('admin/settings/apply_for_role', '', '', 'user_access', 'a:1:{i:0;s:25:"administer apply for role";}', 'drupal_get_form', 'a:1:{i:0;s:28:"apply_for_role_settings_form";}', 7, 3, '', 'admin/settings/apply_for_role', 'Apply for role administration', 't', '', 6, '', 'Administer which roles users can apply for.', '', 0, 'sites/all/modules/apply_for_role/apply_for_role.admin.inc'),
('admin/settings/calais', '', '', 'user_access', 'a:1:{i:0;s:21:"administer calais api";}', 'drupal_get_form', 'a:1:{i:0;s:25:"calais_api_admin_settings";}', 7, 3, '', 'admin/settings/calais', 'Calais configuration', 't', '', 6, '', 'Configurations for Calais', '', 0, ''),
('admin/settings/calais/calais-api', '', '', 'user_access', 'a:1:{i:0;s:21:"administer calais api";}', 'drupal_get_form', 'a:1:{i:0;s:25:"calais_api_admin_settings";}', 15, 4, 'admin/settings/calais', 'admin/settings/calais', 'Calais API Settings', 't', '', 136, '', 'Configurations for Calais API', '', 0, ''),
('admin/settings/calais/calais-node', '', '', 'user_access', 'a:1:{i:0;s:17:"administer calais";}', 'drupal_get_form', 'a:1:{i:0;s:21:"calais_admin_settings";}', 15, 4, 'admin/settings/calais', 'admin/settings/calais', 'Calais Node Settings', 't', '', 128, '', 'Configurations for Calais Integration with content nodes', '', 0, ''),
('admin/settings/calais/calais-tagmods', '', '', 'user_access', 'a:1:{i:0;s:17:"administer calais";}', 'drupal_get_form', 'a:1:{i:0;s:23:"calais_tagmods_settings";}', 15, 4, 'admin/settings/calais', 'admin/settings/calais', 'Calais Tag Modifications', 't', '', 128, '', 'Configurations for Calais Tag Modifications', '', 0, ''),
('admin/settings/calais/geo', '', '', 'user_access', 'a:1:{i:0;s:21:"administer calais geo";}', 'drupal_get_form', 'a:1:{i:0;s:24:"calais_geo_settings_form";}', 15, 4, '', 'admin/settings/calais/geo', 'Calais Geo Settings', 't', '', 6, '', 'Configuration for Calais based Geomapping.', '', 0, 'sites/all/modules/opencalais/contrib/calais_geo/calais_geo.admin.inc'),
('admin/settings/calais/queue', '', '', 'user_access', 'a:1:{i:0;s:17:"administer calais";}', 'calais_bulk_process', 'a:0:{}', 15, 4, 'admin/settings/calais', 'admin/settings/calais', 'Calais Bulk Processing', 't', '', 128, '', 'Submits all Nodes of a specific type to Calais for processing', '', 10, ''),
('admin/settings/calais/queue/clear/%', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:17:"administer calais";}', 'drupal_get_form', 'a:2:{i:0;s:26:"calais_queue_clear_confirm";i:1;i:5;}', 62, 6, '', 'admin/settings/calais/queue/clear/%', 'Clear Calais Bulk Queue', 't', '', 4, '', 'Clears the bulk queue for a particular content type', '', 0, ''),
('admin/settings/calais/queue/enqueue/%', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:17:"administer calais";}', 'calais_enqueue', 'a:1:{i:0;i:5;}', 62, 6, '', 'admin/settings/calais/queue/enqueue/%', 'Calais Queue Processing', 't', '', 4, '', 'Submits all Content of the specified type to Calais for processing', '', 0, ''),
('admin/settings/casetracker', '', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'drupal_get_form', 'a:1:{i:0;s:20:"casetracker_settings";}', 7, 3, '', 'admin/settings/casetracker', 'Case Tracker', 't', '', 6, '', 'Configure the various Case Tracker options with these settings.', '', 0, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/settings', '', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'drupal_get_form', 'a:1:{i:0;s:20:"casetracker_settings";}', 15, 4, 'admin/settings/casetracker', 'admin/settings/casetracker', 'Settings', 't', '', 136, '', '', '', -10, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/states', '', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'casetracker_case_state_overview', 'a:0:{}', 15, 4, 'admin/settings/casetracker', 'admin/settings/casetracker', 'Case states', 't', '', 128, '', 'Add, edit and delete Case States, Types and Priorities', '', 0, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/states/add', '', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'drupal_get_form', 'a:1:{i:0;s:27:"casetracker_case_state_edit";}', 31, 5, 'admin/settings/casetracker/states', 'admin/settings/casetracker', 'Add case state', 't', '', 128, '', '', '', 0, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/states/delete/%', 'a:1:{i:5;s:27:"casetracker_case_state_load";}', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'drupal_get_form', 'a:2:{i:0;s:37:"casetracker_case_state_confirm_delete";i:1;i:5;}', 62, 6, '', 'admin/settings/casetracker/states/delete/%', 'Delete case state', 't', '', 4, '', '', '', 0, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/states/edit/%', 'a:1:{i:5;s:27:"casetracker_case_state_load";}', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'drupal_get_form', 'a:2:{i:0;s:27:"casetracker_case_state_edit";i:1;i:5;}', 62, 6, '', 'admin/settings/casetracker/states/edit/%', 'Edit case state', 't', '', 4, '', '', '', 0, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/casetracker/states/list', '', '', 'user_access', 'a:1:{i:0;s:23:"administer case tracker";}', 'casetracker_case_state_overview', 'a:0:{}', 31, 5, 'admin/settings/casetracker/states', 'admin/settings/casetracker', 'Overview', 't', '', 136, '', 'Add, edit and delete Case States, Types and Priorities', '', -10, 'sites/all/modules/casetracker/casetracker_admin.inc'),
('admin/settings/ckeditor', '', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'ckeditor_admin_main', 'a:0:{}', 7, 3, '', 'admin/settings/ckeditor', 'CKEditor', 't', '', 6, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/add', '', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:1:{i:0;s:27:"ckeditor_admin_profile_form";}', 15, 4, '', 'admin/settings/ckeditor/add', 'Add new CKEditor profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/addg', '', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:2:{i:0;s:34:"ckeditor_admin_global_profile_form";i:1;s:3:"add";}', 15, 4, '', 'admin/settings/ckeditor/addg', 'Add CKEditor Global profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/clone/%', 'a:1:{i:4;s:21:"ckeditor_profile_load";}', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:2:{i:0;s:33:"ckeditor_admin_profile_clone_form";i:1;i:4;}', 30, 5, '', 'admin/settings/ckeditor/clone/%', 'Clone CKEditor profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/delete/%', 'a:1:{i:4;s:21:"ckeditor_profile_load";}', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:2:{i:0;s:34:"ckeditor_admin_profile_delete_form";i:1;i:4;}', 30, 5, '', 'admin/settings/ckeditor/delete/%', 'Delete CKEditor profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/edit/%', 'a:1:{i:4;s:21:"ckeditor_profile_load";}', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:2:{i:0;s:27:"ckeditor_admin_profile_form";i:1;i:4;}', 30, 5, '', 'admin/settings/ckeditor/edit/%', 'Edit CKEditor profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/ckeditor/editg', '', '', 'user_access', 'a:1:{i:0;s:19:"administer ckeditor";}', 'drupal_get_form', 'a:2:{i:0;s:34:"ckeditor_admin_global_profile_form";i:1;s:4:"edit";}', 15, 4, '', 'admin/settings/ckeditor/editg', 'Edit CKEditor Global profile', 't', '', 4, '', 'Configure the rich text editor.', '', 0, 'sites/all/modules/ckeditor/includes/ckeditor.admin.inc'),
('admin/settings/clean-urls', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"system_clean_url_settings";}', 7, 3, '', 'admin/settings/clean-urls', 'Clean URLs', 't', '', 6, '', 'Enable or disable clean URLs for your site.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/clean-urls/check', '', '', '1', 'a:0:{}', 'drupal_json', 'a:1:{i:0;a:1:{s:6:"status";b:1;}}', 15, 4, '', 'admin/settings/clean-urls/check', 'Clean URL check', 't', '', 4, '', '', '', 0, ''),
('admin/settings/conference', '', '', 'user_access', 'a:1:{i:0;s:21:"administer conference";}', 'drupal_get_form', 'a:1:{i:0;s:25:"conference_admin_settings";}', 7, 3, '', 'admin/settings/conference', 'Conference settings', 't', '', 6, '', 'Set up content types, roles, messages etc. relevant to paper submission, review & decision.', '', 0, 'sites/medicineandhealthukm.com/modules/conference/conference.admin.inc'),
('admin/settings/dashboard', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'drupal_get_form', 'a:1:{i:0;s:23:"dashboard_settings_form";}', 7, 3, '', 'admin/settings/dashboard', 'Dashboard', 't', '', 6, '', 'Administer general dashboard settings.', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/add/modal', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_wizard_modal', 'a:0:{}', 31, 5, '', 'admin/settings/dashboard/add/modal', '', 't', '', 6, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/ajax/autocomplete/block', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_autocomplete_block', 'a:0:{}', 63, 6, '', 'admin/settings/dashboard/ajax/autocomplete/block', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/ajax/autocomplete/node', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_autocomplete_node', 'a:0:{}', 63, 6, '', 'admin/settings/dashboard/ajax/autocomplete/node', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/ajax/autocomplete/tag', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_autocomplete_tag', 'a:0:{}', 63, 6, '', 'admin/settings/dashboard/ajax/autocomplete/tag', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/default', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'drupal_get_form', 'a:1:{i:0;s:23:"dashboard_settings_form";}', 15, 4, 'admin/settings/dashboard', 'admin/settings/dashboard', 'General', 't', '', 136, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/widget/%/delete', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'drupal_get_form', 'a:2:{i:0;s:39:"dashboard_default_widget_delete_confirm";i:1;i:4;}', 61, 6, '', 'admin/settings/dashboard/widget/%/delete', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/widget/%/edit', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_wizard_modal', 'a:0:{}', 61, 6, '', 'admin/settings/dashboard/widget/%/edit', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/dashboard/widgets', '', '', 'user_access', 'a:1:{i:0;s:29:"administer dashboard defaults";}', 'dashboard_overview', 'a:0:{}', 15, 4, 'admin/settings/dashboard', 'admin/settings/dashboard', 'Widgets', 't', '', 128, '', 'Shows all available widgets on a the site.', '', 0, 'sites/all/modules/dashboard/dashboard.admin.inc'),
('admin/settings/date-time', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"system_date_time_settings";}', 7, 3, '', 'admin/settings/date-time', 'Date and time', 't', '', 6, '', 'Settings for how Drupal displays date and time, as well as the system''s default timezone.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/date-time/configure', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"system_date_time_settings";}', 15, 4, 'admin/settings/date-time', 'admin/settings/date-time', 'Date and time', 't', '', 136, '', 'Settings for how Drupal displays date and time, as well as the system''s default timezone.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/date-time/delete/%', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:32:"date_api_delete_format_type_form";i:1;i:4;}', 30, 5, '', 'admin/settings/date-time/delete/%', 'Delete date format type', 't', '', 4, '', 'Allow users to delete a configured date format type.', '', 0, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:26:"date_api_date_formats_form";}', 15, 4, 'admin/settings/date-time', 'admin/settings/date-time', 'Formats', 't', '', 128, '', 'Allow users to configure date formats', '', 1, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats/add', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:30:"date_api_add_date_formats_form";}', 31, 5, 'admin/settings/date-time/formats', 'admin/settings/date-time', 'Add format', 't', '', 128, '', 'Allow users to add additional date formats.', '', 3, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats/configure', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:26:"date_api_date_formats_form";}', 31, 5, 'admin/settings/date-time/formats', 'admin/settings/date-time', 'Configure', 't', '', 136, '', 'Allow users to configure date formats', '', 1, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats/custom', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'date_api_configure_custom_date_formats', 'a:0:{}', 31, 5, 'admin/settings/date-time/formats', 'admin/settings/date-time', 'Custom formats', 't', '', 128, '', 'Allow users to configure custom date formats.', '', 2, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats/delete/%', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:27:"date_api_delete_format_form";i:1;i:5;}', 62, 6, '', 'admin/settings/date-time/formats/delete/%', 'Delete date format', 't', '', 4, '', 'Allow users to delete a configured date format.', '', 0, 'sites/all/modules/date/date_api.admin.inc'),
('admin/settings/date-time/formats/lookup', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'date_api_date_time_lookup', 'a:0:{}', 31, 5, '', 'admin/settings/date-time/formats/lookup', 'Date and time lookup', 't', '', 4, '', '', '', 0, ''),
('admin/settings/date-time/locale', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:23:"date_locale_format_form";}', 15, 4, 'admin/settings/date-time', 'admin/settings/date-time', 'Locale date settings', 't', '', 128, '', 'Configure date formats for each locale', '', 1, ''),
('admin/settings/date-time/lookup', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_date_time_lookup', 'a:0:{}', 15, 4, '', 'admin/settings/date-time/lookup', 'Date and time lookup', 't', '', 4, '', '', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/date_popup', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:19:"date_popup_settings";}', 7, 3, '', 'admin/settings/date_popup', 'Date Popup Configuration', 't', '', 6, '', 'Allows the user to configure the Date Popup settings.', '', 0, ''),
('admin/settings/devel', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:20:"devel_admin_settings";}', 7, 3, '', 'admin/settings/devel', 'Devel settings', 't', '', 6, '', 'Helper functions, pages, and blocks to assist Drupal developers. The devel blocks can be managed via the <a href="/admin/build/block">block administration</a> page.', '', 0, ''),
('admin/settings/document_cloud', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:29:"document_cloud_admin_settings";}', 7, 3, '', 'admin/settings/document_cloud', 'DocumentCloud Settings', 't', '', 6, '', 'Configure the DocumentCloud settings for your site', '', 0, ''),
('admin/settings/download_count', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:29:"download_count_admin_settings";}', 7, 3, '', 'admin/settings/download_count', 'Download count settings', 't', '', 6, '', 'Tracks file downloads for files stored in the drupal files table.', '', 0, 'sites/all/modules/download_count/includes/download_count.admin.inc'),
('admin/settings/download_count/general', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:29:"download_count_admin_settings";}', 15, 4, 'admin/settings/download_count', 'admin/settings/download_count', 'General', 't', '', 136, '', '', '', 1, 'sites/all/modules/download_count/includes/download_count.admin.inc'),
('admin/settings/download_count/statistics', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:40:"download_count_statistics_admin_settings";}', 15, 4, 'admin/settings/download_count', 'admin/settings/download_count', 'Statistics', 't', '', 128, '', 'Tracks file downloads for files stored in the drupal files table.', '', 2, 'sites/all/modules/download_count/includes/download_count_statistics.admin.inc'),
('admin/settings/email', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:20:"email_admin_settings";}', 7, 3, '', 'admin/settings/email', 'CCK Email Contact Form Settings', 't', '', 6, '', 'Administer flood control settings for email contact forms', '', 0, ''),
('admin/settings/error-reporting', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:31:"system_error_reporting_settings";}', 7, 3, '', 'admin/settings/error-reporting', 'Error reporting', 't', '', 6, '', 'Control how Drupal deals with errors including 403/404 errors as well as PHP error reporting.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/file-system', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:27:"system_file_system_settings";}', 7, 3, '', 'admin/settings/file-system', 'File system', 't', '', 6, '', 'Tell Drupal where to store uploaded files and how they are accessed.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/file-system/settings', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:27:"system_file_system_settings";}', 15, 4, 'admin/settings/file-system', 'admin/settings/file-system', 'Settings', 't', '', 136, '', '', '', -10, 'sites/all/modules/transliteration/system.admin.inc'),
('admin/settings/file-system/transliteration', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:27:"transliteration_retroactive";}', 15, 4, 'admin/settings/file-system', 'admin/settings/file-system', 'Transliteration', 't', '', 128, '', 'Convert existing file names to US-ASCII.', '', 10, 'sites/all/modules/transliteration/transliteration.admin.inc'),
('admin/settings/filters', '', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'drupal_get_form', 'a:1:{i:0;s:21:"filter_admin_overview";}', 7, 3, '', 'admin/settings/filters', 'Input formats', 't', '', 6, '', 'Configure how content input by users is filtered, including allowed HTML tags. Also allows enabling of module-provided filters.', '', 0, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/%', 'a:1:{i:3;s:18:"filter_format_load";}', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'filter_admin_format_page', 'a:1:{i:0;i:3;}', 14, 4, '', 'admin/settings/filters/%', '', 'filter_admin_format_title', 'a:1:{i:0;i:3;}', 4, '', '', '', 0, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/%/configure', 'a:1:{i:3;s:18:"filter_format_load";}', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'filter_admin_configure_page', 'a:1:{i:0;i:3;}', 29, 5, 'admin/settings/filters/%', 'admin/settings/filters/%', 'Configure', 't', '', 128, '', '', '', 1, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/%/edit', 'a:1:{i:3;s:18:"filter_format_load";}', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'filter_admin_format_page', 'a:1:{i:0;i:3;}', 29, 5, 'admin/settings/filters/%', 'admin/settings/filters/%', 'Edit', 't', '', 136, '', '', '', 0, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/%/order', 'a:1:{i:3;s:18:"filter_format_load";}', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'filter_admin_order_page', 'a:1:{i:0;i:3;}', 29, 5, 'admin/settings/filters/%', 'admin/settings/filters/%', 'Rearrange', 't', '', 128, '', '', '', 2, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/add', '', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'filter_admin_format_page', 'a:0:{}', 15, 4, 'admin/settings/filters', 'admin/settings/filters', 'Add input format', 't', '', 128, '', '', '', 1, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/delete', '', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'drupal_get_form', 'a:1:{i:0;s:19:"filter_admin_delete";}', 15, 4, '', 'admin/settings/filters/delete', 'Delete input format', 't', '', 4, '', '', '', 0, 'modules/filter/filter.admin.inc'),
('admin/settings/filters/list', '', '', 'user_access', 'a:1:{i:0;s:18:"administer filters";}', 'drupal_get_form', 'a:1:{i:0;s:21:"filter_admin_overview";}', 15, 4, 'admin/settings/filters', 'admin/settings/filters', 'List', 't', '', 136, '', '', '', 0, 'modules/filter/filter.admin.inc'),
('admin/settings/flickrapi', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:24:"flickrapi_admin_settings";}', 7, 3, '', 'admin/settings/flickrapi', 'Flickr API Settings', 't', '', 6, '', 'Configure Flickr API credentials.', '', 0, ''),
('admin/settings/gmap', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:19:"gmap_admin_settings";}', 7, 3, '', 'admin/settings/gmap', 'GMap', 't', '', 6, '', 'Configure GMap settings', '', 0, 'sites/all/modules/gmap/gmap_settings_ui.inc'),
('admin/settings/image-toolkit', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:29:"system_image_toolkit_settings";}', 7, 3, '', 'admin/settings/image-toolkit', 'Image toolkit', 't', '', 6, '', 'Choose which image toolkit to use if you have installed optional toolkits.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/imageapi', '', '', 'user_access', 'a:1:{i:0;s:19:"administer imageapi";}', 'drupal_get_form', 'a:1:{i:0;s:17:"imageapi_settings";}', 7, 3, '', 'admin/settings/imageapi', 'ImageAPI', 't', '', 6, '', 'Configure ImageAPI.', '', 0, ''),
('admin/settings/imageapi/config', '', '', 'user_access', 'a:1:{i:0;s:19:"administer imageapi";}', 'drupal_get_form', 'a:1:{i:0;s:25:"imageapi_gd_settings_form";}', 15, 4, 'admin/settings/imageapi', 'admin/settings/imageapi', 'Configure', 't', '', 128, '', '', '', 0, ''),
('admin/settings/imageapi/config/imageapi_gd', '', '', 'user_access', 'a:1:{i:0;s:19:"administer imageapi";}', 'drupal_get_form', 'a:1:{i:0;s:25:"imageapi_gd_settings_form";}', 31, 5, 'admin/settings/imageapi/config', 'admin/settings/imageapi', '@name', 't', 'a:1:{s:5:"@name";s:12:"ImageAPI GD2";}', 136, '', '', '', 0, ''),
('admin/settings/imageapi/list', '', '', 'user_access', 'a:1:{i:0;s:19:"administer imageapi";}', 'drupal_get_form', 'a:1:{i:0;s:17:"imageapi_settings";}', 15, 4, 'admin/settings/imageapi', 'admin/settings/imageapi', 'List', 't', '', 136, '', '', '', -1, ''),
('admin/settings/imagecrop', '', '', 'user_access', 'a:1:{i:0;s:20:"administer imagecrop";}', 'drupal_get_form', 'a:1:{i:0;s:18:"imagecrop_settings";}', 7, 3, '', 'admin/settings/imagecrop', 'Imagecache javascript crop', 't', '', 6, '', '', '', 0, 'sites/all/modules/imagecrop/imagecrop.admin.inc'),
('admin/settings/imce', '', '', 'user_access', 'a:1:{i:0;s:28:"administer imce(execute PHP)";}', 'imce_admin', 'a:0:{}', 7, 3, '', 'admin/settings/imce', 'IMCE', 't', '', 6, '', 'Control how your image/file browser works.', '', 0, 'sites/all/modules/imce/inc/imce.admin.inc'),
('admin/settings/imce/profile', '', '', 'user_access', 'a:1:{i:0;s:28:"administer imce(execute PHP)";}', 'imce_profile_operations', 'a:0:{}', 15, 4, '', 'admin/settings/imce/profile', 'Add new profile', 't', '', 4, '', '', '', 0, 'sites/all/modules/imce/inc/imce.admin.inc'),
('admin/settings/jquery_update', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:22:"jquery_update_settings";}', 7, 3, '', 'admin/settings/jquery_update', 'jQuery Update', 't', '', 6, '', 'Configure settings for jQuery Update module.', '', 0, ''),
('admin/settings/language', '', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:2:{i:0;s:15:"drupal_get_form";i:1;s:30:"locale_languages_overview_form";}', 7, 3, '', 'admin/settings/language', 'Languages', 't', '', 6, '', 'Configure languages for content and the user interface.', '', 0, ''),
('admin/settings/language/add', '', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:1:{i:0;s:27:"locale_languages_add_screen";}', 15, 4, 'admin/settings/language', 'admin/settings/language', 'Add language', 't', '', 128, '', '', '', 5, ''),
('admin/settings/language/configure', '', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:2:{i:0;s:15:"drupal_get_form";i:1;s:31:"locale_languages_configure_form";}', 15, 4, 'admin/settings/language', 'admin/settings/language', 'Configure', 't', '', 128, '', '', '', 10, ''),
('admin/settings/language/delete/%', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:3:{i:0;s:15:"drupal_get_form";i:1;s:28:"locale_languages_delete_form";i:2;i:4;}', 30, 5, '', 'admin/settings/language/delete/%', 'Confirm', 't', '', 4, '', '', '', 0, ''),
('admin/settings/language/edit/%', 'a:1:{i:4;N;}', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:3:{i:0;s:15:"drupal_get_form";i:1;s:26:"locale_languages_edit_form";i:2;i:4;}', 30, 5, '', 'admin/settings/language/edit/%', 'Edit language', 't', '', 4, '', '', '', 0, ''),
('admin/settings/language/overview', '', '', 'user_access', 'a:1:{i:0;s:20:"administer languages";}', 'locale_inc_callback', 'a:2:{i:0;s:15:"drupal_get_form";i:1;s:30:"locale_languages_overview_form";}', 15, 4, 'admin/settings/language', 'admin/settings/language', 'List', 't', '', 136, '', '', '', 0, ''),
('admin/settings/language/update', '', '', 'user_access', 'a:1:{i:0;s:19:"translate interface";}', 'drupal_get_form', 'a:1:{i:0;s:31:"l10n_update_admin_settings_form";}', 15, 4, 'admin/settings/language', 'admin/settings/language', 'Translation updates', 't', '', 128, '', 'Automatic update configuration', '', 20, 'sites/all/modules/l10n_update/l10n_update.admin.inc'),
('admin/settings/lightbox2', '', '', 'user_access', 'a:1:{i:0;s:20:"administer lightbox2";}', 'lightbox2_settings_page', 'a:0:{}', 7, 3, '', 'admin/settings/lightbox2', 'Lightbox2', 't', '', 6, '', 'Allows the user to configure the lightbox2 settings', '', 0, 'sites/all/modules/lightbox2/lightbox2.admin.inc'),
('admin/settings/lightbox2/automatic', '', '', 'user_access', 'a:1:{i:0;s:20:"administer lightbox2";}', 'drupal_get_form', 'a:1:{i:0;s:43:"lightbox2_auto_image_handling_settings_form";}', 15, 4, 'admin/settings/lightbox2', 'admin/settings/lightbox2', 'Automatic image handling', 't', '', 128, '', 'Allows the user to configure the lightbox2 automatic image handling settings', '', 3, 'sites/all/modules/lightbox2/lightbox2.admin.inc'),
('admin/settings/lightbox2/general', '', '', 'user_access', 'a:1:{i:0;s:20:"administer lightbox2";}', 'drupal_get_form', 'a:1:{i:0;s:31:"lightbox2_general_settings_form";}', 15, 4, 'admin/settings/lightbox2', 'admin/settings/lightbox2', 'General', 't', '', 136, '', 'Allows the user to configure the lightbox2 settings', '', 0, 'sites/all/modules/lightbox2/lightbox2.admin.inc'),
('admin/settings/lightbox2/html_content', '', '', 'user_access', 'a:1:{i:0;s:20:"administer lightbox2";}', 'drupal_get_form', 'a:1:{i:0;s:30:"lightbox2_iframe_settings_form";}', 15, 4, 'admin/settings/lightbox2', 'admin/settings/lightbox2', 'HTML Content', 't', '', 128, '', 'Allows the user to configure the lightbox2 HTML content functionality.', '', 2, 'sites/all/modules/lightbox2/lightbox2.admin.inc'),
('admin/settings/lightbox2/slideshow', '', '', 'user_access', 'a:1:{i:0;s:20:"administer lightbox2";}', 'drupal_get_form', 'a:1:{i:0;s:33:"lightbox2_slideshow_settings_form";}', 15, 4, 'admin/settings/lightbox2', 'admin/settings/lightbox2', 'Slideshow', 't', '', 128, '', 'Allows the user to configure the lightbox2 slideshow functionality', '', 1, 'sites/all/modules/lightbox2/lightbox2.admin.inc'),
('admin/settings/logging', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'system_logging_overview', 'a:0:{}', 7, 3, '', 'admin/settings/logging', 'Logging and alerts', 't', '', 6, '', 'Settings for logging and alerts modules. Various modules can route Drupal''s system events to different destination, such as syslog, database, email, ...etc.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/mimemail', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:23:"mimemail_admin_settings";}', 7, 3, '', 'admin/settings/mimemail', 'Mime Mail', 't', '', 6, '', 'HTML E-mail settings', '', 0, 'sites/all/modules/mimemail/includes/mimemail.admin.inc'),
('admin/settings/mollom', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'mollom_admin_form_list', 'a:0:{}', 7, 3, '', 'admin/settings/mollom', 'Mollom', 't', '', 6, '', 'Mollom is a web service that helps you manage your community.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/add', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:27:"mollom_admin_configure_form";}', 15, 4, 'admin/settings/mollom', 'admin/settings/mollom', 'Add form', 't', '', 128, '', '', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/blacklist', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:27:"mollom_admin_blacklist_form";}', 15, 4, 'admin/settings/mollom', 'admin/settings/mollom', 'Blacklists', 't', '', 128, '', 'Configure blacklists.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/blacklist/delete', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:29:"mollom_admin_blacklist_delete";}', 31, 5, '', 'admin/settings/mollom/blacklist/delete', 'Delete', 't', '', 4, '', '', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/blacklist/profanity', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:2:{i:0;s:27:"mollom_admin_blacklist_form";i:1;i:4;}', 31, 5, 'admin/settings/mollom/blacklist', 'admin/settings/mollom', 'Profanity', 't', '', 128, '', 'Configure profanity blacklist entries.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/blacklist/spam', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:27:"mollom_admin_blacklist_form";}', 31, 5, 'admin/settings/mollom/blacklist', 'admin/settings/mollom', 'Spam', 't', '', 136, '', 'Configure spam blacklist entries.', '', -10, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/blacklist/unwanted', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:2:{i:0;s:27:"mollom_admin_blacklist_form";i:1;i:4;}', 31, 5, 'admin/settings/mollom/blacklist', 'admin/settings/mollom', 'Unwanted', 't', '', 128, '', 'Configure unwanted blacklist entries.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/forms', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'mollom_admin_form_list', 'a:0:{}', 15, 4, 'admin/settings/mollom', 'admin/settings/mollom', 'Forms', 't', '', 136, '', '', '', -10, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/manage/%', 'a:1:{i:4;s:16:"mollom_form_load";}', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:2:{i:0;s:27:"mollom_admin_configure_form";i:1;i:4;}', 30, 5, '', 'admin/settings/mollom/manage/%', 'Configure', 't', '', 6, '', '', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/settings', '', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:1:{i:0;s:21:"mollom_admin_settings";}', 15, 4, 'admin/settings/mollom', 'admin/settings/mollom', 'Settings', 't', '', 128, '', 'Configure Mollom keys and global settings.', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/mollom/unprotect/%', 'a:1:{i:4;s:16:"mollom_form_load";}', '', 'user_access', 'a:1:{i:0;s:17:"administer mollom";}', 'drupal_get_form', 'a:2:{i:0;s:27:"mollom_admin_unprotect_form";i:1;i:4;}', 30, 5, '', 'admin/settings/mollom/unprotect/%', 'Unprotect form', 't', '', 4, '', '', '', 0, 'sites/all/modules/mollom/mollom.admin.inc'),
('admin/settings/morelikethis', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:21:"morelikethis_settings";}', 7, 3, '', 'admin/settings/morelikethis', 'More Like This settings', 't', '', 6, '', 'Configuration for More Like This.', '', 0, 'sites/all/modules/morelikethis/morelikethis.admin.inc'),
('admin/settings/morelikethis/flickr', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:28:"morelikethis_flickr_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'Flickr', 't', '', 128, '', '', '', 0, ''),
('admin/settings/morelikethis/general', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:21:"morelikethis_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'General', 't', '', 136, '', '', '', -1, 'sites/all/modules/morelikethis/morelikethis.admin.inc'),
('admin/settings/morelikethis/googlevideo', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:33:"morelikethis_googlevideo_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'Google Video', 't', '', 128, '', '', '', 0, ''),
('admin/settings/morelikethis/taxonomy', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:30:"morelikethis_taxonomy_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'Taxonomy', 't', '', 128, '', '', '', 0, ''),
('admin/settings/morelikethis/yboss', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:27:"morelikethis_yboss_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'Yahoo! BOSS', 't', '', 128, '', '', '', 0, ''),
('admin/settings/morelikethis/ybossimg', '', '', 'user_access', 'a:1:{i:0;s:23:"administer morelikethis";}', 'drupal_get_form', 'a:1:{i:0;s:30:"morelikethis_ybossimg_settings";}', 15, 4, 'admin/settings/morelikethis', 'admin/settings/morelikethis', 'Yahoo! BOSS Images', 't', '', 128, '', '', '', 0, ''),
('admin/settings/node-form-rearrange', '', '', 'user_access', 'a:1:{i:0;s:24:"administer content types";}', 'drupal_get_form', 'a:1:{i:0;s:28:"node_form_rearrange_settings";}', 7, 3, '', 'admin/settings/node-form-rearrange', 'Node Form Rearrange settings', 't', '', 6, '', '', '', 0, ''),
('admin/settings/openpublish', '', '', 'user_access', 'a:1:{i:0;s:12:"set api keys";}', 'drupal_get_form', 'a:1:{i:0;s:24:"openpublish_api_key_form";}', 7, 3, '', 'admin/settings/openpublish', 'OpenPublish', 't', '', 6, '', 'Openpublish-related settings.', '', 0, ''),
('admin/settings/openpublish/api-keys', '', '', 'user_access', 'a:1:{i:0;s:12:"set api keys";}', 'drupal_get_form', 'a:1:{i:0;s:24:"openpublish_api_key_form";}', 15, 4, 'admin/settings/openpublish', 'admin/settings/openpublish', 'OpenPublish API Keys', 't', '', 136, '', '', '', 0, ''),
('admin/settings/openpublish/clear-help', '', '', 'user_access', 'a:1:{i:0;s:30:"access openpublish admin pages";}', 'openpublish_administration_clear_help', 'a:0:{}', 15, 4, '', 'admin/settings/openpublish/clear-help', 'Clear help message', 't', '', 4, '', '', '', 0, ''),
('admin/settings/openpublish/content', '', '', 'user_access', 'a:1:{i:0;s:30:"access openpublish admin pages";}', '_openpublish_administration_content_menu_page', 'a:0:{}', 15, 4, 'admin/settings/openpublish', 'admin/settings/openpublish', 'OpenPublish Content', 't', '', 128, '', '', '', 0, ''),
('admin/settings/openpublish/export-perms', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', '_openpublish_administration_export_perms', 'a:0:{}', 15, 4, '', 'admin/settings/openpublish/export-perms', 'Export System Permissions', 't', '', 4, '', '', '', 0, ''),
('admin/settings/paging', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:15:"paging_settings";}', 7, 3, '', 'admin/settings/paging', 'Paging', 't', '', 6, '', 'Enable or disable paging, configure separator string, toggle automatic paging and more for each content types.', '', 0, ''),
('admin/settings/performance', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:27:"system_performance_settings";}', 7, 3, '', 'admin/settings/performance', 'Performance', 't', '', 6, '', 'Enable or disable page caching for anonymous users and set CSS and JS bandwidth optimization options.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/popular_terms', '', '', 'user_access', 'a:1:{i:0;s:19:"administer taxonomy";}', 'drupal_get_form', 'a:1:{i:0;s:26:"_op_popular_terms_settings";}', 7, 3, 'admin/settings', 'admin/settings', 'Popular Terms Settings', 't', '', 128, '', '', '', 0, ''),
('admin/settings/premium', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:16:"premium_settings";}', 7, 3, '', 'admin/settings/premium', 'Premium', 't', '', 6, '', 'Settings for access control to premium content.', '', 0, ''),
('admin/settings/private_download', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:27:"private_download_admin_form";i:1;N;}', 7, 3, '', 'admin/settings/private_download', 'Private Download', 't', '', 6, '', 'Manage private download configuration.', '', 0, ''),
('admin/settings/rdf', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:18:"rdf_admin_settings";}', 7, 3, '', 'admin/settings/rdf', 'RDF settings', 't', '', 6, '', 'Settings for the RDF API.', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/contexts', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_contexts', 'a:0:{}', 15, 4, '', 'admin/settings/rdf/contexts', 'Contexts', 't', '', 6, '', '', '', 40, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/contexts/list', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_contexts', 'a:0:{}', 31, 5, 'admin/settings/rdf/contexts', 'admin/settings/rdf/contexts', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc');
INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
('admin/settings/rdf/feeds', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_feeds', 'a:0:{}', 15, 4, '', 'admin/settings/rdf/feeds', 'Feeds', 't', '', 6, '', '', '', 20, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/feeds/edit/%', 'a:1:{i:5;N;}', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:2:{i:0;s:14:"rdf_admin_feed";i:1;i:5;}', 62, 6, '', 'admin/settings/rdf/feeds/edit/%', 'Configure RDF feed', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/feeds/list', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_feeds', 'a:0:{}', 31, 5, 'admin/settings/rdf/feeds', 'admin/settings/rdf/feeds', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/mappings', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_mappings', 'a:0:{}', 15, 4, '', 'admin/settings/rdf/mappings', 'Mappings', 't', '', 6, '', '', '', 10, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/mappings/list', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_mappings', 'a:0:{}', 31, 5, 'admin/settings/rdf/mappings', 'admin/settings/rdf/mappings', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/namespaces', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_namespaces', 'a:0:{}', 15, 4, '', 'admin/settings/rdf/namespaces', 'Namespaces', 't', '', 6, '', '', '', 30, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/namespaces/add', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_db_admin_ns_edit', 'a:0:{}', 31, 5, 'admin/settings/rdf/namespaces', 'admin/settings/rdf/namespaces', 'Add namespace', 't', '', 128, '', '', '', 1, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/namespaces/delete', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:22:"rdf_db_admin_ns_delete";}', 31, 5, '', 'admin/settings/rdf/namespaces/delete', 'Delete namespace', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/namespaces/edit', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_db_admin_ns_edit', 'a:0:{}', 31, 5, '', 'admin/settings/rdf/namespaces/edit', 'Edit namespace', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/namespaces/list', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'rdf_admin_namespaces', 'a:0:{}', 31, 5, 'admin/settings/rdf/namespaces', 'admin/settings/rdf/namespaces', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/repositories', '', '', 'user_access', 'a:1:{i:0;s:27:"administer RDF repositories";}', 'rdf_admin_repositories', 'a:0:{}', 15, 4, '', 'admin/settings/rdf/repositories', 'Repositories', 't', '', 6, '', '', '', 50, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/repositories/list', '', '', 'user_access', 'a:1:{i:0;s:27:"administer RDF repositories";}', 'rdf_admin_repositories', 'a:0:{}', 31, 5, 'admin/settings/rdf/repositories', 'admin/settings/rdf/repositories', 'List', 't', '', 136, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/repositories/rdf/add', '', '', 'user_access', 'a:1:{i:0;s:27:"administer RDF repositories";}', 'rdf_db_admin_db_edit', 'a:0:{}', 63, 6, 'admin/settings/rdf/repositories', 'admin/settings/rdf/repositories', 'Add local repository', 't', '', 128, '', '', '', 1, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/repositories/rdf/delete', '', '', 'user_access', 'a:1:{i:0;s:27:"administer RDF repositories";}', 'drupal_get_form', 'a:1:{i:0;s:22:"rdf_db_admin_db_delete";}', 63, 6, '', 'admin/settings/rdf/repositories/rdf/delete', 'Delete local repository', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/rdf/repositories/rdf/edit', '', '', 'user_access', 'a:1:{i:0;s:27:"administer RDF repositories";}', 'rdf_db_admin_db_edit', 'a:0:{}', 63, 6, '', 'admin/settings/rdf/repositories/rdf/edit', 'Edit local repository', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('admin/settings/scheduler', '', '', 'user_access', 'a:1:{i:0;s:20:"administer scheduler";}', 'drupal_get_form', 'a:1:{i:0;s:15:"scheduler_admin";}', 7, 3, '', 'admin/settings/scheduler', 'Scheduler module settings', 't', '', 6, '', 'Allows site admins to configure scheduler.', '', 0, ''),
('admin/settings/scheduler/cron', '', '', '1', 'a:1:{i:0;s:20:"administer scheduler";}', '_scheduler_manual_cron_run', 'a:0:{}', 15, 4, '', 'admin/settings/scheduler/cron', 'Manual scheduler cron run', 't', '', 4, '', 'Allows site admins to run schedulers cron function manually.', '', 0, ''),
('admin/settings/search', '', '', 'user_access', 'a:1:{i:0;s:17:"administer search";}', 'drupal_get_form', 'a:1:{i:0;s:21:"search_admin_settings";}', 7, 3, '', 'admin/settings/search', 'Search settings', 't', '', 6, '', 'Configure relevance settings for search and other indexing options', '', 0, 'modules/search/search.admin.inc'),
('admin/settings/search/wipe', '', '', 'user_access', 'a:1:{i:0;s:17:"administer search";}', 'drupal_get_form', 'a:1:{i:0;s:19:"search_wipe_confirm";}', 15, 4, '', 'admin/settings/search/wipe', 'Clear index', 't', '', 4, '', '', '', 0, 'modules/search/search.admin.inc'),
('admin/settings/site-information', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:32:"system_site_information_settings";}', 7, 3, '', 'admin/settings/site-information', 'Site information', 't', '', 6, '', 'Change basic site information, such as the site name, slogan, e-mail address, mission, front page and more.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/site-maintenance', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:32:"system_site_maintenance_settings";}', 7, 3, '', 'admin/settings/site-maintenance', 'Site maintenance', 't', '', 6, '', 'Take the site off-line for maintenance or bring it back online.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/strongarm', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:20:"strongarm_admin_form";}', 7, 3, '', 'admin/settings/strongarm', 'Strongarm', 't', '', 6, '', 'Manage Drupal variable settings that have been strongarmed.', '', 0, 'sites/all/modules/strongarm/strongarm.admin.inc'),
('admin/settings/swftools', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'system_admin_menu_block_page', 'a:0:{}', 7, 3, '', 'admin/settings/swftools', 'SWF Tools', 't', '', 6, '', 'Settings to control how SWF Tools integrates with Adobe Flash related methods and tools like video players, MP3 players and image viewers.', '', 0, 'modules/system/system.admin.inc'),
('admin/settings/swftools/cck', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:23:"swftools_admin_cck_form";}', 15, 4, '', 'admin/settings/swftools/cck', 'CCK formatters', 't', '', 6, '', 'Additional settings to control how SWF Tools should interact with CCK.', '', 0, 'sites/all/modules/swftools/swftools.admin.inc'),
('admin/settings/swftools/embed', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:25:"swftools_admin_embed_form";}', 15, 4, '', 'admin/settings/swftools/embed', 'Embedding settings', 't', '', 6, '', 'Set the embedding method that SWF Tools should use, and configure embedding defaults.', '', -2, 'sites/all/modules/swftools/swftools.admin.inc'),
('admin/settings/swftools/flowplayer3', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:26:"flowplayer3_admin_settings";}', 15, 4, '', 'admin/settings/swftools/flowplayer3', 'FlowPlayer 3', 't', '', 6, '', 'Settings for <a href="http://flowplayer.org/">FlowPlayer 3</a>.', '', 0, 'sites/all/modules/swftools/flowplayer3/flowplayer3.admin.inc'),
('admin/settings/swftools/generic', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:27:"swftools_admin_generic_form";}', 15, 4, '', 'admin/settings/swftools/generic', 'Generic players', 't', '', 6, '', 'Basic Flash players that ship with SWF Tools', '', -1, ''),
('admin/settings/swftools/handling', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:28:"swftools_admin_handling_form";}', 15, 4, '', 'admin/settings/swftools/handling', 'File handling', 't', '', 6, '', 'Configure how SWF Tools should handle different types of file.', '', -1, 'sites/all/modules/swftools/swftools.admin.inc'),
('admin/settings/swftools/onepixelout', '', '', 'user_access', 'a:1:{i:0;s:16:"administer flash";}', 'drupal_get_form', 'a:1:{i:0;s:22:"onepixelout_admin_form";}', 15, 4, '', 'admin/settings/swftools/onepixelout', '1 Pixel Out', 't', '', 6, '', 'Settings for <a href="http://www.1pixelout.net/code/audio-player-wordpress-plugin/">1 Pixel Out MP3 Player</a>.', '', 0, 'sites/all/modules/swftools/onepixelout/onepixelout.admin.inc'),
('admin/settings/tabs', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:19:"tabs_admin_settings";}', 7, 3, '', 'admin/settings/tabs', 'Tabs', 't', '', 6, '', 'Configuration for tabs', '', 0, 'sites/all/modules/tabs/tabs.admin.inc'),
('admin/settings/token_array', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"token_array_settings_form";}', 7, 3, '', 'admin/settings/token_array', 'Array token settings', 't', '', 6, '', '', '', 0, 'sites/all/modules/token_array/token_array.admin.inc'),
('admin/settings/uploadpath', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:25:"uploadpath_admin_settings";}', 7, 3, '', 'admin/settings/uploadpath', 'File upload paths', 't', '', 6, '', 'Configure a prefix to apply to the path of uploaded files.', '', 0, 'sites/all/modules/uploadpath/uploadpath.admin.inc'),
('admin/settings/uploads', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'drupal_get_form', 'a:1:{i:0;s:21:"upload_admin_settings";}', 7, 3, '', 'admin/settings/uploads', 'File uploads', 't', '', 6, '', 'Control how files may be attached to content.', '', 0, 'modules/upload/upload.admin.inc'),
('admin/user', '', '', 'admin_landing_page_access', 'a:1:{i:0;s:10:"admin/user";}', 'system_admin_menu_block_page', 'a:0:{}', 3, 2, '', 'admin/user', 'User management', 't', '', 6, '', 'Manage your site''s users, groups and access to site features.', 'left', 0, 'modules/system/system.admin.inc'),
('admin/user/apply_for_role', '', '', 'user_access', 'a:1:{i:0;s:25:"approve role applications";}', 'drupal_get_form', 'a:1:{i:0;s:25:"apply_for_role_admin_form";}', 7, 3, '', 'admin/user/apply_for_role', 'Manage role applications', 't', '', 6, '', 'View, approve and delete role applications.', '', 0, 'sites/all/modules/apply_for_role/apply_for_role.admin.inc'),
('admin/user/apply_for_role/approve/%/%', 'a:2:{i:4;s:9:"user_load";i:5;N;}', '', 'user_access', 'a:1:{i:0;s:25:"approve role applications";}', 'drupal_get_form', 'a:3:{i:0;s:27:"apply_for_role_approve_form";i:1;i:4;i:2;i:5;}', 60, 6, '', 'admin/user/apply_for_role/approve/%/%', 'Approve role application', 't', '', 4, '', '', '', 0, 'sites/all/modules/apply_for_role/apply_for_role.admin.inc'),
('admin/user/apply_for_role/deny/%/%', 'a:2:{i:4;s:9:"user_load";i:5;N;}', '', 'user_access', 'a:1:{i:0;s:25:"approve role applications";}', 'drupal_get_form', 'a:3:{i:0;s:24:"apply_for_role_deny_form";i:1;i:4;i:2;i:5;}', 60, 6, '', 'admin/user/apply_for_role/deny/%/%', 'Deny role application', 't', '', 4, '', '', '', 0, 'sites/all/modules/apply_for_role/apply_for_role.admin.inc'),
('admin/user/apply_for_role/remove/%/%', 'a:2:{i:4;s:9:"user_load";i:5;N;}', '', 'user_access', 'a:1:{i:0;s:25:"approve role applications";}', 'drupal_get_form', 'a:3:{i:0;s:26:"apply_for_role_remove_form";i:1;i:4;i:2;i:5;}', 60, 6, '', 'admin/user/apply_for_role/remove/%/%', 'Remove role application', 't', '', 4, '', '', '', 0, 'sites/all/modules/apply_for_role/apply_for_role.admin.inc'),
('admin/user/autoassignrole', '', '', 'user_access', 'a:1:{i:0;s:25:"administer autoassignrole";}', 'drupal_get_form', 'a:1:{i:0;s:25:"autoassignrole_admin_form";}', 7, 3, '', 'admin/user/autoassignrole', 'Auto assign role', 't', '', 6, '', 'Designate a role to assign all new users to.', '', 0, 'sites/all/modules/autoassignrole/autoassignrole-admin.inc'),
('admin/user/autoassignrole/autocomplete/node', '', '', 'user_access', 'a:1:{i:0;s:25:"administer autoassignrole";}', 'autoassignrole_autocomplete_node', 'a:0:{}', 31, 5, '', 'admin/user/autoassignrole/autocomplete/node', 'Node autocomplete', 't', '', 4, '', '', '', 0, 'sites/all/modules/autoassignrole/autoassignrole-admin.inc'),
('admin/user/login_destination', '', '', 'user_access', 'a:1:{i:0;s:28:"administer login destination";}', 'drupal_get_form', 'a:1:{i:0;s:32:"login_destination_admin_settings";}', 7, 3, '', 'admin/user/login_destination', 'Login Destination', 't', '', 6, '', 'Control where users are redirected to, once they login.', '', 0, ''),
('admin/user/permissions', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'drupal_get_form', 'a:1:{i:0;s:15:"user_admin_perm";}', 7, 3, '', 'admin/user/permissions', 'Permissions', 't', '', 6, '', 'Determine access to features by selecting permissions for roles.', '', 0, 'modules/user/user.admin.inc'),
('admin/user/profile', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'drupal_get_form', 'a:1:{i:0;s:22:"profile_admin_overview";}', 7, 3, '', 'admin/user/profile', 'Profiles', 't', '', 6, '', 'Create customizable fields for your users.', '', 0, 'modules/profile/profile.admin.inc'),
('admin/user/profile/add', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'drupal_get_form', 'a:1:{i:0;s:18:"profile_field_form";}', 15, 4, '', 'admin/user/profile/add', 'Add field', 't', '', 4, '', '', '', 0, 'modules/profile/profile.admin.inc'),
('admin/user/profile/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'profile_admin_settings_autocomplete', 'a:0:{}', 15, 4, '', 'admin/user/profile/autocomplete', 'Profile category autocomplete', 't', '', 4, '', '', '', 0, 'modules/profile/profile.admin.inc'),
('admin/user/profile/delete', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'drupal_get_form', 'a:1:{i:0;s:20:"profile_field_delete";}', 15, 4, '', 'admin/user/profile/delete', 'Delete field', 't', '', 4, '', '', '', 0, 'modules/profile/profile.admin.inc'),
('admin/user/profile/edit', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'drupal_get_form', 'a:1:{i:0;s:18:"profile_field_form";}', 15, 4, '', 'admin/user/profile/edit', 'Edit field', 't', '', 4, '', '', '', 0, 'modules/profile/profile.admin.inc'),
('admin/user/profile_locality', '', '', 'user_access', 'a:1:{i:0;s:29:"administer site configuration";}', 'profile_locality_admin', 'a:0:{}', 7, 3, '', 'admin/user/profile_locality', 'Profile Locality', 't', '', 6, '', 'Configure your Profile Locality preferences and field assignments.', '', 0, ''),
('admin/user/roles', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'drupal_get_form', 'a:1:{i:0;s:19:"user_admin_new_role";}', 7, 3, '', 'admin/user/roles', 'Roles', 't', '', 6, '', 'List, edit, or add user roles.', '', 0, 'modules/user/user.admin.inc'),
('admin/user/roles/edit', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'drupal_get_form', 'a:1:{i:0;s:15:"user_admin_role";}', 15, 4, '', 'admin/user/roles/edit', 'Edit role', 't', '', 4, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'user_admin_access', 'a:0:{}', 7, 3, '', 'admin/user/rules', 'Access rules', 't', '', 6, '', 'List and create rules to disallow usernames, e-mail addresses, and IP addresses.', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules/add', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'user_admin_access_add', 'a:0:{}', 15, 4, 'admin/user/rules', 'admin/user/rules', 'Add rule', 't', '', 128, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules/check', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'user_admin_access_check', 'a:0:{}', 15, 4, 'admin/user/rules', 'admin/user/rules', 'Check rules', 't', '', 128, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules/delete', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'drupal_get_form', 'a:1:{i:0;s:32:"user_admin_access_delete_confirm";}', 15, 4, '', 'admin/user/rules/delete', 'Delete rule', 't', '', 4, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules/edit', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'user_admin_access_edit', 'a:0:{}', 15, 4, '', 'admin/user/rules/edit', 'Edit rule', 't', '', 4, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/rules/list', '', '', 'user_access', 'a:1:{i:0;s:22:"administer permissions";}', 'user_admin_access', 'a:0:{}', 15, 4, 'admin/user/rules', 'admin/user/rules', 'List', 't', '', 136, '', '', '', -10, 'modules/user/user.admin.inc'),
('admin/user/settings', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'drupal_get_form', 'a:1:{i:0;s:19:"user_admin_settings";}', 7, 3, '', 'admin/user/settings', 'User settings', 't', '', 6, '', 'Configure default behavior of users, including registration requirements, e-mails, and user pictures.', '', 0, 'modules/user/user.admin.inc'),
('admin/user/user', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'user_admin', 'a:1:{i:0;s:4:"list";}', 7, 3, '', 'admin/user/user', 'Users', 't', '', 6, '', 'List, add, and edit users.', '', 0, 'modules/user/user.admin.inc'),
('admin/user/user/advuser', '', '', 'user_access', 'a:1:{i:0;s:14:"access advuser";}', 'advuser_admin', 'a:1:{i:0;s:4:"list";}', 15, 4, 'admin/user/user', 'admin/user/user', 'Advanced', 't', '', 128, '', 'List, add, edit and email users.', '', 0, 'sites/all/modules/advuser/advuser_filters.inc'),
('admin/user/user/create', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'user_admin', 'a:1:{i:0;s:6:"create";}', 15, 4, 'admin/user/user', 'admin/user/user', 'Add user', 't', '', 128, '', '', '', 0, 'modules/user/user.admin.inc'),
('admin/user/user/list', '', '', 'user_access', 'a:1:{i:0;s:16:"administer users";}', 'user_admin', 'a:1:{i:0;s:4:"list";}', 15, 4, 'admin/user/user', 'admin/user/user', 'List', 't', '', 136, '', '', '', -10, 'modules/user/user.admin.inc'),
('admin/user/userprotect', '', '', 'user_access', 'a:1:{i:0;s:22:"administer userprotect";}', 'drupal_get_form', 'a:1:{i:0;s:27:"userprotect_protected_users";}', 7, 3, '', 'admin/user/userprotect', 'User Protect', 't', '', 6, '', 'Protect inidividual users and/or roles from editing operations.', '', 0, ''),
('admin/user/userprotect/administrator_bypass', '', '', 'user_access', 'a:1:{i:0;s:22:"administer userprotect";}', 'drupal_get_form', 'a:1:{i:0;s:32:"userprotect_administrator_bypass";}', 15, 4, 'admin/user/userprotect', 'admin/user/userprotect', 'Administrator bypass', 't', '', 128, '', '', '', 3, ''),
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('admin/user/userprotect/protected_users', '', '', 'user_access', 'a:1:{i:0;s:22:"administer userprotect";}', 'drupal_get_form', 'a:1:{i:0;s:27:"userprotect_protected_users";}', 15, 4, 'admin/user/userprotect', 'admin/user/userprotect', 'Protected users', 't', '', 136, '', '', '', 1, ''),
('admin/user/userprotect/protection_defaults', '', '', 'user_access', 'a:1:{i:0;s:22:"administer userprotect";}', 'drupal_get_form', 'a:1:{i:0;s:31:"userprotect_protection_defaults";}', 15, 4, 'admin/user/userprotect', 'admin/user/userprotect', 'Protection defaults', 't', '', 128, '', '', '', 4, ''),
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('devel/php', '', '', 'user_access', 'a:1:{i:0;s:16:"execute php code";}', 'drupal_get_form', 'a:1:{i:0;s:18:"devel_execute_form";}', 3, 2, '', 'devel/php', 'Execute PHP Code', 't', '', 6, '', 'Execute some PHP code', '', 0, ''),
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('documents', '', '', 'user_access', 'a:1:{i:0;s:28:"view documentcloud documents";}', '_document_cloud_view', 'a:1:{i:0;i:1;}', 1, 1, '', 'documents', '', 't', '', 4, '', '', '', 0, ''),
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('find/articles/byissue', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:15:"articles_search";i:1;s:6:"page_3";}', 7, 3, '', 'find/articles/byissue', '', 't', '', 4, '', '', '', 4, ''),
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('find/articles/issue', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:15:"articles_search";i:1;s:6:"page_1";}', 7, 3, 'find/articles', 'find/articles', 'Advanced Search', 't', '', 136, '', 'Search articles by issue, author, title, abstract or keywords', '', 1, ''),
('flag', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'flag_page', 'a:0:{}', 1, 1, '', 'flag', 'Flag', 't', '', 4, '', '', '', 0, ''),
('flag/confirm', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'drupal_get_form', 'a:1:{i:0;s:12:"flag_confirm";}', 3, 2, '', 'flag/confirm', 'Flag confirm', 't', '', 4, '', '', '', 0, ''),
('fulltext/%', 'a:1:{i:1;a:1:{s:14:"views_arg_load";a:3:{i:0;s:20:"image_gallery_images";i:1;s:6:"page_1";i:2;s:6:"%index";}}}', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:2:{i:1;i:1;i:2;i:2;}}}}', 'views_page', 'a:3:{i:0;s:20:"image_gallery_images";i:1;s:6:"page_1";i:2;i:1;}', 2, 2, '', 'fulltext/%', '', 't', '', 4, '', '', '', 0, ''),
('help/%/%', 'a:2:{i:1;N;i:2;N;}', '', 'user_access', 'a:1:{i:0;s:24:"view advanced help topic";}', 'advanced_help_topic_page', 'a:2:{i:0;i:1;i:1;i:2;}', 4, 3, '', 'help/%/%', '', 't', '', 4, '', '', '', 0, ''),
('ibrowser', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:8:"archivez";i:1;s:6:"page_1";}', 1, 1, '', 'ibrowser', '', 't', '', 4, '', '', '', 0, ''),
('imagecrop/docrop', '', '', 'user_access', 'a:1:{i:0;s:24:"crop images with toolbox";}', 'imagecrop_docrop', 'a:0:{}', 3, 2, '', 'imagecrop/docrop', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/imagecrop/imagecrop.admin.inc'),
('imagecrop/showcrop', '', '', 'user_access', 'a:1:{i:0;s:24:"crop images with toolbox";}', 'imagecrop_showcrop', 'a:0:{}', 3, 2, '', 'imagecrop/showcrop', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/imagecrop/imagecrop.admin.inc'),
('imce', '', '', 'imce_access', 'a:0:{}', 'imce', 'a:0:{}', 1, 1, '', 'imce', 'File browser', 't', '', 4, '', '', '', 0, 'sites/all/modules/imce/inc/imce.page.inc'),
('issue-browser', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:12:"date_browser";i:1;s:4:"page";}', 1, 1, '', 'issue-browser', '', 't', '', 4, '', '', '', 0, ''),
('l10n_client/save', '', '', '0', 'a:0:{}', 'l10n_update_client_save_string', 'a:0:{}', 3, 2, '', 'l10n_client/save', '', 't', '', 6, '', '', '', 0, ''),
('logout', '', '', 'user_is_logged_in', 'a:0:{}', 'user_logout', 'a:0:{}', 1, 1, '', 'logout', 'Log out', 't', '', 6, '', '', '', 10, 'modules/user/user.pages.inc'),
('mimemail', '', '', '0', 'a:0:{}', 'mimemail_post', 'a:0:{}', 1, 1, '', 'mimemail', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/mimemail/includes/mimemail.incoming.inc'),
('mollom/captcha/%/%', 'a:2:{i:2;N;i:3;N;}', '', '_mollom_access', 'a:0:{}', 'mollom_captcha_js', 'a:2:{i:0;i:2;i:1;i:3;}', 12, 4, '', 'mollom/captcha/%/%', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/mollom/mollom.pages.inc'),
('mollom/report/%/%', 'a:2:{i:2;N;i:3;N;}', '', 'mollom_report_access', 'a:2:{i:0;i:2;i:1;i:3;}', 'drupal_get_form', 'a:3:{i:0;s:18:"mollom_report_form";i:1;i:2;i:2;i:3;}', 12, 4, '', 'mollom/report/%/%', 'Report to Mollom', 't', '', 4, '', '', '', 0, 'sites/all/modules/mollom/mollom.pages.inc'),
('most-used-terms', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', '_op_popular_terms', 'a:0:{}', 1, 1, '', 'most-used-terms', 'Most Used Terms', 't', '', 128, '', '', '', 0, ''),
('multimedia', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:10:"multimedia";i:1;s:6:"page_1";}', 1, 1, '', 'multimedia', '', 't', '', 4, '', '', '', 0, ''),
('my_download_count', '', '', 'user_access', 'a:1:{i:0;s:24:"view own download counts";}', 'download_count_view_page', 'a:1:{i:0;s:17:"my_download_count";}', 1, 1, '', 'my_download_count', 'My Download Count', 't', '', 6, '', '', '', 0, 'sites/all/modules/download_count//includes/download_count.pages.inc'),
('news', '', '', 'views_access', 'a:2:{i:0;b:1;i:1;b:1;}', 'views_page', 'a:2:{i:0;s:7:"archive";i:1;a:2:{i:0;s:4:"page";i:1;s:6:"page_1";}}', 1, 1, '', 'news', '', 't', '', 4, '', '', '', 0, ''),
('node', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'node_page_default', 'a:0:{}', 1, 1, '', 'node', 'Content', 't', '', 4, '', '', '', 0, ''),
('node/%', 'a:1:{i:1;s:9:"node_load";}', '', 'node_access', 'a:2:{i:0;s:4:"view";i:1;i:1;}', 'page_manager_node_view', 'a:1:{i:0;i:1;}', 2, 2, '', 'node/%', '', 'node_page_title', 'a:1:{i:0;i:1;}', 4, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/node_view.inc'),
('node/%/calais', 'a:1:{i:1;s:9:"node_load";}', '', 'calais_access', 'a:1:{i:0;i:1;}', 'drupal_get_form', 'a:2:{i:0;s:20:"calais_keywords_form";i:1;i:1;}', 5, 3, 'node/%', 'node/%', 'Calais', 't', '', 128, '', '', '', 2, ''),
('node/%/calais/rdf', 'a:1:{i:1;N;}', '', 'user_access', 'a:1:{i:0;s:17:"access calais rdf";}', 'calais_get_rdf', 'a:1:{i:0;i:1;}', 11, 4, '', 'node/%/calais/rdf', 'Calais', 't', '', 4, '', '', '', 0, ''),
('node/%/delete', 'a:1:{i:1;s:9:"node_load";}', '', 'node_access', 'a:2:{i:0;s:6:"delete";i:1;i:1;}', 'drupal_get_form', 'a:2:{i:0;s:19:"node_delete_confirm";i:1;i:1;}', 5, 3, '', 'node/%/delete', 'Delete', 't', '', 4, '', '', '', 1, 'modules/node/node.pages.inc'),
('node/%/devel', 'a:1:{i:1;s:9:"node_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'devel_load_object', 'a:2:{i:0;i:1;i:1;s:4:"node";}', 5, 3, 'node/%', 'node/%', 'Devel', 't', '', 128, '', '', '', 100, ''),
('node/%/devel/load', 'a:1:{i:1;s:9:"node_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'devel_load_object', 'a:2:{i:0;i:1;i:1;s:4:"node";}', 11, 4, 'node/%/devel', 'node/%', 'Dev load', 't', '', 136, '', '', '', 0, ''),
('node/%/devel/render', 'a:1:{i:1;s:9:"node_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'devel_render_object', 'a:2:{i:0;s:4:"node";i:1;i:1;}', 11, 4, 'node/%/devel', 'node/%', 'Dev render', 't', '', 128, '', '', '', 10, ''),
('node/%/devel/token', 'a:1:{i:1;s:9:"node_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'token_devel_token_object', 'a:2:{i:0;s:4:"node";i:1;i:1;}', 11, 4, 'node/%/devel', 'node/%', 'Tokens', 't', '', 128, '', '', '', 5, 'sites/all/modules/token/token.pages.inc'),
('node/%/edit', 'a:1:{i:1;s:9:"node_load";}', '', 'node_access', 'a:2:{i:0;s:6:"update";i:1;i:1;}', 'node_page_edit', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Edit', 't', '', 128, '', '', '', 1, 'modules/node/node.pages.inc'),
('node/%/nodequeue', 'a:1:{i:1;s:9:"node_load";}', '', 'nodequeue_node_tab_access', 'a:1:{i:0;i:1;}', 'nodequeue_node_tab', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', '@tab', 't', 'a:1:{s:4:"@tab";s:9:"Nodequeue";}', 128, '', '', '', 5, ''),
('node/%/panel_content', 'a:1:{i:1;s:9:"node_load";}', '', 'panels_node_edit_node', 'a:1:{i:0;i:1;}', 'panels_node_edit_content', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Panel content', 't', '', 128, '', '', '', 3, ''),
('node/%/panel_layout', 'a:1:{i:1;s:9:"node_load";}', '', 'panels_node_edit_node', 'a:1:{i:0;i:1;}', 'panels_node_edit_layout', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Panel layout', 't', '', 128, '', '', '', 2, ''),
('node/%/rdf', 'a:1:{i:1;s:9:"node_load";}', '', 'rdf_menu_access_node', 'a:2:{i:0;s:4:"view";i:1;i:1;}', 'rdf_export_node', 'a:1:{i:0;i:1;}', 5, 3, '', 'node/%/rdf', 'RDF', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('node/%/relationships', 'a:1:{i:1;s:9:"node_load";}', '', 'noderelationships_backref_access', 'a:1:{i:0;i:1;}', 'noderelationships_backref_page', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Relationships', 't', '', 128, '', '', '', 10, 'sites/all/modules/noderelationships/noderelationships.pages.inc'),
('node/%/repeats', 'a:1:{i:1;s:9:"node_load";}', '', 'date_repeat_node', 'a:1:{i:0;i:1;}', 'date_repeat_page', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Repeats', 't', '', 128, '', '', '', 0, ''),
('node/%/results', 'a:1:{i:1;s:9:"node_load";}', '', '_poll_menu_access', 'a:3:{i:0;i:1;i:1;s:14:"access content";i:2;b:1;}', 'poll_results', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Results', 't', '', 128, '', '', '', 3, 'modules/poll/poll.pages.inc'),
('node/%/revisions', 'a:1:{i:1;s:9:"node_load";}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'diff_diffs_overview', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Revisions', 't', '', 128, '', '', '', 2, 'sites/all/modules/diff/diff.pages.inc'),
('node/%/revisions/%/delete', 'a:2:{i:1;a:1:{s:9:"node_load";a:1:{i:0;i:3;}}i:3;N;}', '', 'diff_node_revision_access', 'a:2:{i:0;i:1;i:1;s:6:"delete";}', 'drupal_get_form', 'a:2:{i:0;s:28:"node_revision_delete_confirm";i:1;i:1;}', 21, 5, '', 'node/%/revisions/%/delete', 'Delete earlier revision', 't', '', 4, '', '', '', 0, 'modules/node/node.pages.inc'),
('node/%/revisions/%/revert', 'a:2:{i:1;a:1:{s:9:"node_load";a:1:{i:0;i:3;}}i:3;N;}', '', 'diff_node_revision_access', 'a:2:{i:0;i:1;i:1;s:6:"update";}', 'drupal_get_form', 'a:2:{i:0;s:28:"node_revision_revert_confirm";i:1;i:1;}', 21, 5, '', 'node/%/revisions/%/revert', 'Revert to earlier revision', 't', '', 4, '', '', '', 0, 'modules/node/node.pages.inc'),
('node/%/revisions/%/view', 'a:2:{i:1;a:1:{s:9:"node_load";a:1:{i:0;i:3;}}i:3;N;}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'node_show', 'a:3:{i:0;i:1;i:1;N;i:2;b:1;}', 21, 5, '', 'node/%/revisions/%/view', 'Revisions', 't', '', 4, '', '', '', 0, ''),
('node/%/revisions/diff-inline', 'a:1:{i:1;s:9:"node_load";}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'diff_inline_ahah', 'a:1:{i:0;i:1;}', 11, 4, '', 'node/%/revisions/diff-inline', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/diff/diff.pages.inc'),
('node/%/revisions/list', 'a:1:{i:1;s:9:"node_load";}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'diff_diffs_overview', 'a:0:{}', 11, 4, 'node/%/revisions', 'node/%', 'List revisions', 't', '', 136, '', '', '', 0, 'sites/all/modules/diff/diff.pages.inc'),
('node/%/revisions/view/%/%', 'a:3:{i:1;s:9:"node_load";i:4;N;i:5;N;}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'diff_diffs_show', 'a:3:{i:0;i:1;i:1;i:4;i:2;i:5;}', 44, 6, 'node/%/revisions/list', 'node/%', 'Diff', 't', '', 128, '', '', '', 0, 'sites/all/modules/diff/diff.pages.inc'),
('node/%/revisions/view/latest', 'a:1:{i:1;s:9:"node_load";}', '', 'diff_node_revision_access', 'a:1:{i:0;i:1;}', 'diff_latest', 'a:1:{i:0;i:1;}', 23, 5, 'node/%/revisions/view', 'node/%', 'Show latest diff', 't', '', 128, '', '', '', 0, 'sites/all/modules/diff/diff.pages.inc'),
('node/%/topichub', 'a:1:{i:1;s:9:"node_load";}', '', 'topichubs_access', 'a:1:{i:0;i:1;}', 'drupal_get_form', 'a:2:{i:0;s:21:"topichubs_config_form";i:1;i:1;}', 5, 3, 'node/%', 'node/%', 'Topic Hub Configuration', 't', '', 128, '', '', '', 2, ''),
('node/%/topic_panel_content', 'a:1:{i:1;s:9:"node_load";}', '', 'panels_node_edit_node', 'a:1:{i:0;s:20:"administer topichubs";}', 'topichubs_panels_edit_content', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'TopicHub content', 't', '', 128, '', '', '', 3, '');
INSERT INTO `menu_router` (`path`, `load_functions`, `to_arg_functions`, `access_callback`, `access_arguments`, `page_callback`, `page_arguments`, `fit`, `number_parts`, `tab_parent`, `tab_root`, `title`, `title_callback`, `title_arguments`, `type`, `block_callback`, `description`, `position`, `weight`, `file`) VALUES
('node/%/topic_panel_layout', 'a:1:{i:1;s:9:"node_load";}', '', 'panels_node_edit_node', 'a:1:{i:0;s:20:"administer topichubs";}', 'topichubs_panels_edit_layout', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'TopicHub layout', 't', '', 128, '', '', '', 2, ''),
('node/%/topic_panel_settings', 'a:1:{i:1;s:9:"node_load";}', '', 'panels_node_edit_node', 'a:1:{i:0;s:20:"administer topichubs";}', 'topichubs_panels_edit_layout_settings', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'TopicHub layout settings', 't', '', 128, '', '', '', 2, ''),
('node/%/track', 'a:1:{i:1;s:9:"node_load";}', '', 'user_access', 'a:1:{i:0;s:17:"access statistics";}', 'statistics_node_tracker', 'a:0:{}', 5, 3, 'node/%', 'node/%', 'Track', 't', '', 128, '', '', '', 2, 'modules/statistics/statistics.pages.inc'),
('node/%/translate', 'a:1:{i:1;s:9:"node_load";}', '', '_translation_tab_access', 'a:1:{i:0;i:1;}', 'translation_node_overview', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Translate', 't', '', 128, '', '', '', 2, 'modules/translation/translation.pages.inc'),
('node/%/view', 'a:1:{i:1;s:9:"node_load";}', '', 'node_access', 'a:2:{i:0;s:4:"view";i:1;i:1;}', 'page_manager_node_view', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'View', 't', '', 136, '', '', '', -10, 'sites/all/modules/ctools/page_manager/plugins/tasks/node_view.inc'),
('node/%/votes', 'a:1:{i:1;s:9:"node_load";}', '', '_poll_menu_access', 'a:3:{i:0;i:1;i:1;s:17:"inspect all votes";i:2;b:0;}', 'poll_votes', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Votes', 't', '', 128, '', '', '', 3, 'modules/poll/poll.pages.inc'),
('node/%/workflow', 'a:1:{i:1;s:9:"node_load";}', '', 'workflow_node_tab_access', 'a:1:{i:0;i:1;}', 'workflow_tab_page', 'a:1:{i:0;i:1;}', 5, 3, 'node/%', 'node/%', 'Workflow', 't', '', 128, '', '', '', 2, 'sites/all/modules/workflow/workflow.pages.inc'),
('node/add', '', '', '_node_add_access', 'a:0:{}', 'node_add_page', 'a:0:{}', 3, 2, '', 'node/add', 'Create content', 't', '', 6, '', '', '', 1, 'modules/node/node.pages.inc'),
('node/add/article', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:7:"article";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/article', 'Article', 'check_plain', '', 6, '', 'Articles can be posted to the site in category lists and on the homepage.', '', 0, 'modules/node/node.pages.inc'),
('node/add/audio', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:5:"audio";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/audio', 'Audio', 'check_plain', '', 6, '', 'Audio content can be entered by referencing an external source, or by uploading a local audio file into the CMS.', '', 0, 'modules/node/node.pages.inc'),
('node/add/author', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:6:"author";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/author', 'Author', 'check_plain', '', 6, '', 'Author can be associated with a Drupal user, but can also be a contributing author that does not have Drupal login to your website.', '', 0, 'modules/node/node.pages.inc'),
('node/add/blog', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:4:"blog";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/blog', 'Blog entry', 'check_plain', '', 6, '', 'A <em>blog entry</em> is a single post to an online journal, or <em>blog</em>.', '', 0, 'modules/node/node.pages.inc'),
('node/add/casetracker-basic-case', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:22:"casetracker_basic_case";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/casetracker-basic-case', 'Case', 'check_plain', '', 6, '', 'Open a new case assigned to a particular project.', '', 0, 'modules/node/node.pages.inc'),
('node/add/casetracker-basic-project', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:25:"casetracker_basic_project";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/casetracker-basic-project', 'Project', 'check_plain', '', 6, '', 'Create a basic project for use with Case Tracker.', '', 0, 'modules/node/node.pages.inc'),
('node/add/conference-paper', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:16:"conference_paper";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/conference-paper', 'Manuscript', 'check_plain', '', 6, '', 'This form is used for submitting a manuscript.<br />', '', 0, 'modules/node/node.pages.inc'),
('node/add/conference-review', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:17:"conference_review";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/conference-review', 'Manuscript review', 'check_plain', '', 6, '', 'This form is used for submitting a review of a given manuscript contribution.<br /><br />\r\n\r\nThis review is restricted to the dedicated ''reviewer'' role chosen by the editorial board, and access to the paper is restricted to it''s author and the editor.', '', 0, 'modules/node/node.pages.inc'),
('node/add/editors-choice', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:14:"editors_choice";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/editors-choice', 'Editor''s Choice', 'check_plain', '', 6, '', 'This type of content can be created for display as a block on the Homepage, right sidebar and other sections.', '', 0, 'modules/node/node.pages.inc'),
('node/add/event', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:5:"event";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/event', 'Event', 'check_plain', '', 6, '', 'Events are calendar style content items that are referenced by date.', '', 0, 'modules/node/node.pages.inc'),
('node/add/issue', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:5:"issue";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/issue', 'Issue', 'check_plain', '', 6, '', '', '', 0, 'modules/node/node.pages.inc'),
('node/add/menu', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:4:"menu";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/menu', 'Menu', 'check_plain', '', 6, '', '', '', 0, 'modules/node/node.pages.inc'),
('node/add/news', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:4:"news";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/news', 'News', 'check_plain', '', 6, '', '', '', 0, 'modules/node/node.pages.inc'),
('node/add/op-image', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:8:"op_image";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/op-image', 'Image', 'check_plain', '', 6, '', 'Images can be uploaded into the CMS and used in a variety of capacities.', '', 0, 'modules/node/node.pages.inc'),
('node/add/package', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:7:"package";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/package', 'Package', 'check_plain', '', 6, '', 'News packages are sets of topical information based around one relevant event or topic. Examples might include an election, or coverage of the war in Iraq.', '', 0, 'modules/node/node.pages.inc'),
('node/add/page', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:4:"page";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/page', 'Page', 'check_plain', '', 6, '', 'A <em>page</em>, similar in form to a <em>story</em>, is a simple method for creating and displaying information that rarely changes, such as an "About us" section of a website. By default, a <em>page</em> entry does not allow visitor comments and is not featured on the site''s initial home page.', '', 0, 'modules/node/node.pages.inc'),
('node/add/panel', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:5:"panel";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/panel', 'Panel', 'check_plain', '', 6, '', 'A panel layout broken up into rows and columns.', '', 0, 'modules/node/node.pages.inc'),
('node/add/panel/choose-layout', '', '', 'user_access', 'a:1:{i:0;s:18:"create panel-nodes";}', 'panels_node_add', 'a:0:{}', 15, 4, '', 'node/add/panel/choose-layout', 'Choose layout', 't', '', 4, '', '', '', 0, ''),
('node/add/poll', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:4:"poll";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/poll', 'Poll', 'check_plain', '', 6, '', 'A <em>poll</em> is a question with a set of possible responses. A <em>poll</em>, once created, automatically provides a simple running count of the number of votes received for each response.', '', 0, 'modules/node/node.pages.inc'),
('node/add/profile', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:7:"profile";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/profile', 'Profile', 'check_plain', '', 6, '', 'A user profile built as content.', '', 0, 'modules/node/node.pages.inc'),
('node/add/resource', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:8:"resource";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/resource', 'Resource', 'check_plain', '', 6, '', 'Resources are links, files, documents and other items of interest to readers. This content type is intended to provide a flexible mechanism for providing content that doesn''t fit other content types, and can be differentiated from others via flexible categorization.', '', 0, 'modules/node/node.pages.inc'),
('node/add/slideshow', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:9:"slideshow";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/slideshow', 'Slideshow', 'check_plain', '', 6, '', 'Slideshow is a content type that contains a set of images.', '', 0, 'modules/node/node.pages.inc'),
('node/add/topichub', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:8:"topichub";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/topichub', 'Topic Hub', 'check_plain', '', 6, '', 'These nodes represent a collection of related data pertaining to the same topic.', '', 0, 'modules/node/node.pages.inc'),
('node/add/topichub_panel/choose-layout', '', '', 'user_access', 'a:1:{i:0;s:20:"administer topichubs";}', 'topichubs_panels_add', 'a:0:{}', 15, 4, '', 'node/add/topichub_panel/choose-layout', 'Choose layout', 't', '', 4, '', '', '', 0, ''),
('node/add/video', '', '', 'node_access', 'a:2:{i:0;s:6:"create";i:1;s:5:"video";}', 'node_add', 'a:1:{i:0;i:2;}', 7, 3, '', 'node/add/video', 'Video', 'check_plain', '', 6, '', 'Video content can be entered by referencing an external source, like YouTube, or by uploading a local video file into the CMS.', '', 0, 'modules/node/node.pages.inc'),
('nodequeue/1', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:11:"nodequeue_1";i:1;s:4:"page";}', 3, 2, '', 'nodequeue/1', '', 't', '', 4, '', '', '', 0, ''),
('nodequeue/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:17:"manipulate queues";}', 'nodequeue_autocomplete', 'a:0:{}', 3, 2, '', 'nodequeue/autocomplete', 'Autocomplete', 't', '', 4, '', '', '', 0, ''),
('nodereference/autocomplete', '', '', 'nodereference_autocomplete_access', 'a:1:{i:0;i:2;}', 'nodereference_autocomplete', 'a:0:{}', 3, 2, '', 'nodereference/autocomplete', 'Nodereference autocomplete', 't', '', 4, '', '', '', 0, ''),
('noderelationships/ahah/search', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'noderelationships_noderef_ahah_search', 'a:0:{}', 7, 3, '', 'noderelationships/ahah/search', 'Search and reference', 't', '', 4, '', '', '', 0, 'sites/all/modules/noderelationships/noderelationships.pages.inc'),
('noderelationships/create', '', '', '_node_add_access', 'a:0:{}', 'noderelationships_noderef_page', 'a:1:{i:0;i:1;}', 3, 2, '', 'noderelationships/create', 'Create and reference', 't', '', 4, '', '', '', 0, 'sites/all/modules/noderelationships/noderelationships.pages.inc'),
('noderelationships/noderelationships-backref', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:25:"noderelationships_backref";i:1;s:4:"page";}', 3, 2, '', 'noderelationships/noderelationships-backref', '', 't', '', 4, '', '', '', 0, ''),
('noderelationships/noderelationships-noderef/page-grid', '', '', 'views_access', 'a:2:{i:0;b:1;i:1;b:1;}', 'views_page', 'a:2:{i:0;s:25:"noderelationships_noderef";i:1;a:2:{i:0;s:9:"page_grid";i:1;s:9:"page_grid";}}', 7, 3, '', 'noderelationships/noderelationships-noderef/page-grid', '', 't', '', 4, '', '', '', 0, ''),
('noderelationships/noderelationships-noderef/page-table', '', '', 'views_access', 'a:2:{i:0;b:1;i:1;b:1;}', 'views_page', 'a:2:{i:0;s:25:"noderelationships_noderef";i:1;a:2:{i:0;s:10:"page_table";i:1;s:10:"page_table";}}', 7, 3, '', 'noderelationships/noderelationships-noderef/page-table', '', 't', '', 4, '', '', '', 0, ''),
('noderelationships/search', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'noderelationships_noderef_page', 'a:1:{i:0;i:1;}', 3, 2, '', 'noderelationships/search', 'Search and reference', 't', '', 4, '', '', '', 0, 'sites/all/modules/noderelationships/noderelationships.pages.inc'),
('panels/ajax', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'panels_ajax_router', 'a:0:{}', 3, 2, '', 'panels/ajax', '', 't', '', 4, '', '', '', 0, ''),
('poll', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'poll_page', 'a:0:{}', 1, 1, '', 'poll', 'Polls', 't', '', 20, '', '', '', 0, 'modules/poll/poll.pages.inc'),
('poll/js', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'poll_choice_js', 'a:0:{}', 3, 2, '', 'poll/js', 'Javascript Choice Form', 't', '', 4, '', '', '', 0, ''),
('popular', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:7:"popular";i:1;s:4:"page";}', 1, 1, '', 'popular', 'Popular content', 't', '', 6, '', '', '', 0, ''),
('popular/all', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:7:"popular";i:1;s:4:"page";}', 3, 2, 'popular', 'popular', 'Popular content', 't', '', 136, '', '', '', -1, ''),
('popular/today', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:7:"popular";i:1;s:6:"page_1";}', 3, 2, 'popular', 'popular', 'Today''s popular content', 't', '', 128, '', '', '', 0, ''),
('postal/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'profile_locality_postal_autocomplete', 'a:0:{}', 3, 2, '', 'postal/autocomplete', 'Postal autocomplete', 't', '', 4, '', '', '', 0, ''),
('profile', '', '', 'user_access', 'a:1:{i:0;s:20:"access user profiles";}', 'profile_browse', 'a:0:{}', 1, 1, '', 'profile', 'User list', 't', '', 20, '', '', '', 0, 'modules/profile/profile.pages.inc'),
('profile/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:20:"access user profiles";}', 'profile_autocomplete', 'a:0:{}', 3, 2, '', 'profile/autocomplete', 'Profile autocomplete', 't', '', 4, '', '', '', 0, 'modules/profile/profile.pages.inc'),
('rdf', '', '', 'user_access', 'a:1:{i:0;s:15:"access RDF data";}', 'rdf_export_site', 'a:0:{}', 1, 1, '', 'rdf', 'RDF', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('rdf/autocomplete/class', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'rdf_admin_autocomplete_class', 'a:0:{}', 7, 3, '', 'rdf/autocomplete/class', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('rdf/autocomplete/property', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'rdf_admin_autocomplete_property', 'a:0:{}', 7, 3, '', 'rdf/autocomplete/property', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('rdf/autocomplete/resource', '', '', 'user_access', 'a:1:{i:0;s:19:"administer RDF data";}', 'rdf_admin_autocomplete_resource', 'a:0:{}', 7, 3, '', 'rdf/autocomplete/resource', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.admin.inc'),
('rdf/modules', '', '', 'rdf_menu_access_http', 'a:2:{i:0;s:22:"export enabled modules";i:1;s:22:"import enabled modules";}', 'rdf_schema_export_modules', 'a:0:{}', 3, 2, '', 'rdf/modules', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('rdf/schema', '', '', 'user_access', 'a:1:{i:0;s:15:"access RDF data";}', 'rdf_export', 'a:2:{i:0;s:15:"rdf_schema_data";i:1;s:6:"schema";}', 3, 2, '', 'rdf/schema', 'RDF schema', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('rdf/settings', '', '', 'rdf_menu_access_http', 'a:2:{i:0;s:20:"export site settings";i:1;s:20:"import site settings";}', 'rdf_schema_export_settings', 'a:0:{}', 3, 2, '', 'rdf/settings', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('resources', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:9:"resources";i:1;s:6:"page_1";}', 1, 1, '', 'resources', '', 't', '', 4, '', '', '', 0, ''),
('rss.xml', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'node_feed', 'a:0:{}', 1, 1, '', 'rss.xml', 'RSS feed', 't', '', 4, '', '', '', 0, ''),
('rss/articles', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:8:"articles";i:1;s:6:"feed_1";}', 3, 2, '', 'rss/articles', '', 't', '', 4, '', '', '', 0, ''),
('rss/blogs', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:5:"blogs";i:1;s:6:"feed_1";}', 3, 2, '', 'rss/blogs', '', 't', '', 4, '', '', '', 0, ''),
('rss/events', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:6:"events";i:1;s:6:"feed_1";}', 3, 2, '', 'rss/events', '', 't', '', 4, '', '', '', 0, ''),
('rss/multimedia', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:10:"multimedia";i:1;s:6:"feed_1";}', 3, 2, '', 'rss/multimedia', '', 't', '', 4, '', '', '', 0, ''),
('rss/resources', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:9:"resources";i:1;s:6:"feed_1";}', 3, 2, '', 'rss/resources', '', 't', '', 4, '', '', '', 0, ''),
('scheduler/cron', '', '', '1', 'a:0:{}', '_scheduler_run_cron', 'a:0:{}', 3, 2, '', 'scheduler/cron', 'Light weight cron handler', 't', '', 4, '', 'A light weight cron handler to allow more frequent runs of Schedulers internal cron system', '', 0, ''),
('scheduler/timecheck', '', '', 'user_access', 'a:1:{i:0;s:27:"access administration pages";}', '_scheduler_timecheck', 'a:0:{}', 3, 2, '', 'scheduler/timecheck', 'Test your servers UTC clock', 't', '', 4, '', 'Allows site admin to check their servers internal clock', '', 0, ''),
('search', '', '', 'user_access', 'a:1:{i:0;s:14:"search content";}', 'search_view', 'a:0:{}', 1, 1, '', 'search', 'Search', 't', '', 20, '', '', '', 0, 'modules/search/search.pages.inc'),
('search/advanced_help', '', '', 'user_access', 'a:1:{i:0;s:24:"view advanced help index";}', 'search_view', 'a:1:{i:0;s:13:"advanced_help";}', 3, 2, 'search', 'search', '', 'module_invoke', 'a:4:{i:0;s:13:"advanced_help";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 128, '', '', '', 0, 'modules/search/search.pages.inc'),
('search/advanced_help/%', 'a:1:{i:2;N;}', 'a:1:{i:2;s:16:"menu_tail_to_arg";}', 'user_access', 'a:1:{i:0;s:24:"view advanced help index";}', 'page_manager_search_view', 'a:1:{i:0;s:13:"advanced_help";}', 6, 3, 'search/node/%menu_tail', 'search/node/%menu_tail', '', 'module_invoke', 'a:4:{i:0;s:13:"advanced_help";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 128, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/search.inc'),
('search/node', '', '', '_search_menu', 'a:1:{i:0;s:4:"node";}', 'search_view', 'a:1:{i:0;s:4:"node";}', 3, 2, 'search', 'search', '', 'module_invoke', 'a:4:{i:0;s:4:"node";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 136, '', '', '', -10, 'modules/search/search.pages.inc'),
('search/node/%', 'a:1:{i:2;N;}', 'a:1:{i:2;s:16:"menu_tail_to_arg";}', '_search_menu', 'a:1:{i:0;s:4:"node";}', 'page_manager_search_view', 'a:1:{i:0;s:4:"node";}', 6, 3, 'search/node/%menu_tail', 'search/node/%menu_tail', '', 'module_invoke', 'a:4:{i:0;s:4:"node";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 128, '', '', '', -10, 'sites/all/modules/ctools/page_manager/plugins/tasks/search.inc'),
('search/user', '', '', '_search_menu', 'a:1:{i:0;s:4:"user";}', 'search_view', 'a:1:{i:0;s:4:"user";}', 3, 2, 'search', 'search', '', 'module_invoke', 'a:4:{i:0;s:4:"user";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 128, '', '', '', 0, 'modules/search/search.pages.inc'),
('search/user/%', 'a:1:{i:2;N;}', 'a:1:{i:2;s:16:"menu_tail_to_arg";}', '_search_menu', 'a:1:{i:0;s:4:"user";}', 'page_manager_search_view', 'a:1:{i:0;s:4:"user";}', 6, 3, 'search/node/%menu_tail', 'search/node/%menu_tail', '', 'module_invoke', 'a:4:{i:0;s:4:"user";i:1;s:6:"search";i:2;s:4:"name";i:3;b:1;}', 128, '', '', '', 0, 'sites/all/modules/ctools/page_manager/plugins/tasks/search.inc'),
('sites/medicineandhealthukm.com/files/imagecache', '', '', '1', 'a:0:{}', 'imagecache_cache', 'a:0:{}', 15, 4, '', 'sites/medicineandhealthukm.com/files/imagecache', '', 't', '', 4, '', '', '', 0, ''),
('system/files', '', '', '1', 'a:1:{i:0;s:19:"view uploaded files";}', 'file_download', 'a:0:{}', 3, 2, '', 'system/files', 'File download', 't', '', 4, '', '', '', 0, ''),
('system/files/imagecache', '', '', '1', 'a:0:{}', 'imagecache_cache_private', 'a:0:{}', 7, 3, '', 'system/files/imagecache', '', 't', '', 4, '', '', '', 0, ''),
('system/lightbox2/filter-xss', '', '', '1', 'a:0:{}', 'lightbox2_filter_xss', 'a:0:{}', 7, 3, '', 'system/lightbox2/filter-xss', 'Filter XSS', 't', '', 4, '', '', '', 0, ''),
('taxonomy/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'taxonomy_autocomplete', 'a:0:{}', 3, 2, '', 'taxonomy/autocomplete', 'Autocomplete taxonomy', 't', '', 4, '', '', '', 0, 'modules/taxonomy/taxonomy.pages.inc'),
('taxonomy/term/%', 'a:1:{i:2;N;}', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'taxonomy_term_page', 'a:1:{i:0;i:2;}', 6, 3, '', 'taxonomy/term/%', 'Taxonomy term', 't', '', 4, '', '', '', 0, 'modules/taxonomy/taxonomy.pages.inc'),
('toc', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:7:"journal";i:1;s:6:"page_2";}', 1, 1, '', 'toc', '', 't', '', 4, '', '', '', 0, ''),
('toc/%/%', 'a:2:{i:1;a:1:{s:14:"views_arg_load";a:3:{i:0;s:7:"journal";i:1;s:6:"page_1";i:2;s:6:"%index";}}i:2;a:1:{s:14:"views_arg_load";a:3:{i:0;s:7:"journal";i:1;s:6:"page_1";i:2;s:6:"%index";}}}', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:4:{i:0;s:7:"journal";i:1;s:6:"page_1";i:2;i:1;i:3;i:2;}', 4, 3, '', 'toc/%/%', '', 't', '', 4, '', '', '', 0, ''),
('topic-hubs', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:10:"topic_hubs";i:1;s:6:"page_1";}', 1, 1, '', 'topic-hubs', '', 't', '', 4, '', '', '', 0, ''),
('topichub-detail/most-comments', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:22:"topichub_most_comments";i:1;s:6:"page_1";}', 3, 2, '', 'topichub-detail/most-comments', '', 't', '', 4, '', '', '', 0, ''),
('topichub-detail/most-recent', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:20:"topichub_most_recent";i:1;s:6:"page_1";}', 3, 2, '', 'topichub-detail/most-recent', '', 't', '', 4, '', '', '', 0, ''),
('topichub-detail/most-viewed', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:20:"topichub_most_viewed";i:1;s:6:"page_1";}', 3, 2, '', 'topichub-detail/most-viewed', '', 't', '', 4, '', '', '', 0, ''),
('topichub-detail/recent-comments', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:24:"topichub_recent_comments";i:1;s:6:"page_1";}', 3, 2, '', 'topichub-detail/recent-comments', '', 't', '', 4, '', '', '', 0, ''),
('topics', '', '', 'views_access', 'a:1:{i:0;b:1;}', 'views_page', 'a:2:{i:0;s:16:"content_by_topic";i:1;s:6:"page_1";}', 1, 1, '', 'topics', '', 't', '', 4, '', '', '', 0, ''),
('upload/js', '', '', 'user_access', 'a:1:{i:0;s:12:"upload files";}', 'upload_js', 'a:0:{}', 3, 2, '', 'upload/js', '', 't', '', 4, '', '', '', 0, ''),
('user', '', '', '1', 'a:0:{}', 'user_page', 'a:0:{}', 1, 1, '', 'user', 'User account', 't', '', 4, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/%', 'a:1:{i:1;s:22:"user_uid_optional_load";}', 'a:1:{i:1;s:24:"user_uid_optional_to_arg";}', 'user_view_access', 'a:1:{i:0;i:1;}', 'user_view', 'a:1:{i:0;i:1;}', 2, 2, '', 'user/%', 'My account', 'user_page_title', 'a:1:{i:0;i:1;}', 6, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/%/apply_for_role', 'a:1:{i:1;s:9:"user_load";}', '', 'apply_for_role_apply_access', 'a:1:{i:0;i:1;}', 'drupal_get_form', 'a:2:{i:0;s:25:"apply_for_role_apply_form";i:1;i:1;}', 5, 3, 'user/%', 'user/%', 'Apply for role', 't', '', 128, '', '', '', 0, ''),
('user/%/bio', 'a:1:{i:1;a:1:{s:14:"views_arg_load";a:3:{i:0;s:10:"author_bio";i:1;s:6:"page_1";i:2;s:6:"%index";}}}', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:2:{i:3;i:3;i:5;i:5;}}}}', 'views_page', 'a:3:{i:0;s:10:"author_bio";i:1;s:6:"page_1";i:2;i:1;}', 5, 3, 'user/%', 'user/%', 'Author Profile', 't', '', 128, '', '', '', 0, ''),
('user/%/bookmarks', 'a:1:{i:1;a:1:{s:14:"views_arg_load";a:3:{i:0;s:18:"flag_bookmarks_tab";i:1;s:4:"page";i:2;s:6:"%index";}}}', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:17:"views_check_roles";i:1;a:1:{i:0;a:1:{i:2;s:1:"2";}}}}', 'views_page', 'a:3:{i:0;s:18:"flag_bookmarks_tab";i:1;s:4:"page";i:2;i:1;}', 5, 3, 'user/%', 'user/%', 'Bookmarks', 't', '', 128, '', '', '', 0, ''),
('user/%/contact', 'a:1:{i:1;s:9:"user_load";}', '', '_contact_user_tab_access', 'a:1:{i:0;i:1;}', 'contact_user_page', 'a:1:{i:0;i:1;}', 5, 3, 'user/%', 'user/%', 'Contact', 't', '', 128, '', '', '', 2, 'modules/contact/contact.pages.inc'),
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('user/%/devel', 'a:1:{i:1;s:9:"user_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'devel_load_object', 'a:2:{i:0;i:1;i:1;s:4:"user";}', 5, 3, 'user/%', 'user/%', 'Devel', 't', '', 128, '', '', '', 100, ''),
('user/%/devel/load', 'a:1:{i:1;s:9:"user_load";}', '', 'user_access', 'a:1:{i:0;s:24:"access devel information";}', 'devel_load_object', 'a:2:{i:0;i:1;i:1;s:4:"user";}', 11, 4, 'user/%/devel', 'user/%', 'Dev load', 't', '', 136, '', '', '', 0, ''),
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('user/%/edit', 'a:1:{i:1;a:1:{s:18:"user_category_load";a:2:{i:0;s:4:"%map";i:1;s:6:"%index";}}}', '', 'userprotect_user_edit_access', 'a:1:{i:0;i:1;}', 'user_edit', 'a:1:{i:0;i:1;}', 5, 3, 'user/%', 'user/%', 'Edit', 't', '', 128, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/%/edit/account', 'a:1:{i:1;a:1:{s:18:"user_category_load";a:2:{i:0;s:4:"%map";i:1;s:6:"%index";}}}', '', 'userprotect_user_edit_access', 'a:1:{i:0;i:1;}', 'user_edit', 'a:1:{i:0;i:1;}', 11, 4, 'user/%/edit', 'user/%', 'Account', 't', '', 136, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/%/edit/Personal Info', 'a:1:{i:1;a:1:{s:18:"user_category_load";a:2:{i:0;s:4:"%map";i:1;s:6:"%index";}}}', '', 'profile_category_access', 'a:2:{i:0;i:1;i:1;s:13:"Personal Info";}', 'user_edit', 'a:2:{i:0;i:1;i:1;i:3;}', 11, 4, 'user/%/edit', 'user/%', '', 'check_plain', 'a:1:{i:0;s:13:"Personal Info";}', 128, '', '', '', 3, 'modules/user/user.pages.inc'),
('user/%/imce', 'a:1:{i:1;s:9:"user_load";}', '', 'imce_user_page_access', 'a:1:{i:0;i:1;}', 'imce_user_page', 'a:1:{i:0;i:1;}', 5, 3, 'user/%', 'user/%', 'File browser', 't', '', 128, '', '', '', 10, 'sites/all/modules/imce/inc/imce.page.inc'),
('user/%/rdf', 'a:1:{i:1;s:9:"user_load";}', '', 'rdf_menu_access_user', 'a:1:{i:0;i:1;}', 'rdf_export_user', 'a:1:{i:0;i:1;}', 5, 3, '', 'user/%/rdf', 'RDF', 't', '', 4, '', '', '', 0, 'sites/all/modules/rdf/rdf.pages.inc'),
('user/%/roles', 'a:1:{i:1;s:9:"user_load";}', '', 'role_delegation_access', 'a:0:{}', 'drupal_get_form', 'a:2:{i:0;s:26:"role_delegation_roles_form";i:1;i:1;}', 5, 3, 'user/%', 'user/%', 'Roles', 't', '', 128, '', '', '', 0, ''),
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('user/%/view', 'a:1:{i:1;s:9:"user_load";}', '', 'user_view_access', 'a:1:{i:0;i:1;}', 'user_view', 'a:1:{i:0;i:1;}', 5, 3, 'user/%', 'user/%', 'View', 't', '', 136, '', '', '', -10, 'modules/user/user.pages.inc'),
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('user/login', '', '', 'user_is_anonymous', 'a:0:{}', 'user_page', 'a:0:{}', 3, 2, 'user', 'user', 'Log in', 't', '', 136, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/login/lightbox2', '', '', 'user_is_anonymous', 'a:0:{}', 'lightbox2_login', 'a:0:{}', 7, 3, '', 'user/login/lightbox2', 'Login', 't', '', 4, '', '', '', 0, ''),
('user/password', '', '', 'user_is_anonymous', 'a:0:{}', 'drupal_get_form', 'a:1:{i:0;s:9:"user_pass";}', 3, 2, 'user', 'user', 'Request new password', 't', '', 128, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/register', '', '', 'user_register_access', 'a:0:{}', 'drupal_goto', 'a:1:{i:0;s:20:"user/register/author";}', 3, 2, 'user', 'user', 'Create new account', 't', '', 128, '', '', '', 0, 'modules/user/user.pages.inc'),
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('user/register/author/login', '', '', 'user_is_anonymous', 'a:0:{}', '_autoassignrole_user_login', 'a:0:{}', 15, 4, 'user/register/author', 'user/register/author', 'Log in', 't', '', 128, '', '', '', 0, 'sites/all/modules/autoassignrole/autoassignrole-path.inc'),
('user/register/author/password', '', '', 'user_is_anonymous', 'a:0:{}', '_autoassignrole_user_password', 'a:0:{}', 15, 4, 'user/register/author', 'user/register/author', 'Request new password', 't', '', 128, '', '', '', 0, 'sites/all/modules/autoassignrole/autoassignrole-path.inc'),
('user/register/author/register', '', '', '_autoassignrole_path_access', 'a:0:{}', 'autoassignrole_path', 'a:1:{i:0;s:1:"5";}', 15, 4, 'user/register/author', 'user/register/author', 'Create new account', 't', '', 136, '', '', '', 0, 'sites/all/modules/autoassignrole/autoassignrole-path.inc'),
('user/reset/%/%/%', 'a:3:{i:2;N;i:3;N;i:4;N;}', '', '1', 'a:0:{}', 'drupal_get_form', 'a:4:{i:0;s:15:"user_pass_reset";i:1;i:2;i:2;i:3;i:3;i:4;}', 24, 5, '', 'user/reset/%/%/%', 'Reset password', 't', '', 4, '', '', '', 0, 'modules/user/user.pages.inc'),
('user/timezone', '', '', '1', 'a:0:{}', 'user_timezone', 'a:0:{}', 3, 2, '', 'user/timezone', 'User timezone', 't', '', 4, '', '', '', 0, ''),
('userprotect/delete/%', 'a:1:{i:2;s:9:"user_load";}', '', 'user_access', 'a:1:{i:0;s:22:"administer userprotect";}', 'drupal_get_form', 'a:3:{i:0;s:39:"userprotect_protected_users_delete_form";i:1;i:2;i:2;i:3;}', 6, 3, '', 'userprotect/delete/%', 'Delete protected user', 't', '', 4, '', '', '', 0, ''),
('userreference/autocomplete', '', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'userreference_autocomplete', 'a:0:{}', 3, 2, '', 'userreference/autocomplete', 'Userreference autocomplete', 't', '', 4, '', '', '', 0, ''),
('video-cck/lightbox2/%', 'a:1:{i:2;s:9:"node_load";}', '', 'node_access', 'a:2:{i:0;s:4:"view";i:1;i:2;}', 'lightbox2_emvideo', 'a:1:{i:0;i:2;}', 6, 3, '', 'video-cck/lightbox2/%', '', 't', '', 4, '', '', '', 0, ''),
('views/ajax', '', '', '1', 'a:0:{}', 'views_ajax', 'a:0:{}', 3, 2, '', 'views/ajax', 'Views', 't', '', 4, '', 'Ajax callback for view loading.', '', 0, 'sites/all/modules/views/includes/ajax.inc'),
('views_slideshow_ddblock/js', '', '', '1', 'a:0:{}', 'views_slideshow_ddblock_select_template_ahah', 'a:0:{}', 3, 2, '', 'views_slideshow_ddblock/js', '', 't', '', 4, '', '', '', 0, 'sites/all/modules/views_slideshow_ddblock/views_slideshow_ddblock.views_slideshow.inc'),
('views_slideshow_ddblock_mapping/js/%', 'a:1:{i:2;N;}', '', 'user_access', 'a:1:{i:0;s:14:"access content";}', 'views_slideshow_ddblock_mapping_js', 'a:1:{i:0;i:2;}', 6, 3, '', 'views_slideshow_ddblock_mapping/js/%', '', 't', '', 6, '', '', '', 0, ''),
('workflow', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:16:"views_check_perm";i:1;a:1:{i:0;s:29:"access workflow summary views";}}}', 'views_page', 'a:2:{i:0;s:16:"workflow_summary";i:1;s:6:"page_1";}', 1, 1, '', 'workflow', 'Workflow summary', 't', '', 6, '', '', '', 0, ''),
('workflow/pending', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:16:"views_check_perm";i:1;a:1:{i:0;s:29:"access workflow summary views";}}}', 'views_page', 'a:2:{i:0;s:16:"workflow_pending";i:1;s:6:"page_1";}', 3, 2, 'workflow', 'workflow', 'Pending', 't', '', 128, '', '', '', 0, ''),
('workflow/summary', '', '', 'views_access', 'a:1:{i:0;a:2:{i:0;s:16:"views_check_perm";i:1;a:1:{i:0;s:29:"access workflow summary views";}}}', 'views_page', 'a:2:{i:0;s:16:"workflow_summary";i:1;s:6:"page_1";}', 3, 2, 'workflow', 'workflow', 'Summary', 't', '', 136, '', '', '', -1, '');

-- --------------------------------------------------------

--
-- Table structure for table `mollom`
--

CREATE TABLE IF NOT EXISTS `mollom` (
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  `id` varchar(32) NOT NULL DEFAULT '',
  `session_id` varchar(255) NOT NULL DEFAULT '',
  `form_id` varchar(255) NOT NULL DEFAULT '',
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) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `mollom`
--

INSERT INTO `mollom` (`entity`, `id`, `session_id`, `form_id`, `changed`, `spam`, `quality`, `languages`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `mollom_form`
--

CREATE TABLE IF NOT EXISTS `mollom_form` (
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  `mode` tinyint(4) NOT NULL DEFAULT '0',
  `checks` text,
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--
-- Dumping data for table `mollom_form`
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INSERT INTO `mollom_form` (`form_id`, `mode`, `checks`, `discard`, `enabled_fields`, `module`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `morelikethis`
--

CREATE TABLE IF NOT EXISTS `morelikethis` (
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  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `term` varchar(255) DEFAULT NULL,
  KEY `nid` (`nid`),
  KEY `vid` (`vid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `morelikethis_node_settings`
--

CREATE TABLE IF NOT EXISTS `morelikethis_node_settings` (
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  `nid` int(10) unsigned NOT NULL DEFAULT '0',
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--
-- Dumping data for table `morelikethis_node_settings`
--

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-- --------------------------------------------------------

--
-- Table structure for table `node`
--

CREATE TABLE IF NOT EXISTS `node` (
  `nid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `vid` int(10) unsigned NOT NULL DEFAULT '0',
  `type` varchar(32) NOT NULL DEFAULT '',
  `language` varchar(12) NOT NULL DEFAULT '',
  `title` varchar(255) NOT NULL DEFAULT '',
  `uid` int(11) NOT NULL DEFAULT '0',
  `status` int(11) NOT NULL DEFAULT '1',
  `created` int(11) NOT NULL DEFAULT '0',
  `changed` int(11) NOT NULL DEFAULT '0',
  `comment` int(11) NOT NULL DEFAULT '0',
  `promote` int(11) NOT NULL DEFAULT '0',
  `moderate` int(11) NOT NULL DEFAULT '0',
  `sticky` int(11) NOT NULL DEFAULT '0',
  `tnid` int(10) unsigned NOT NULL DEFAULT '0',
  `translate` int(11) NOT NULL DEFAULT '0',
  PRIMARY KEY (`nid`),
  UNIQUE KEY `vid` (`vid`),
  KEY `node_changed` (`changed`),
  KEY `node_created` (`created`),
  KEY `node_moderate` (`moderate`),
  KEY `node_promote_status` (`promote`,`status`),
  KEY `node_status_type` (`status`,`type`,`nid`),
  KEY `node_title_type` (`title`,`type`(4)),
  KEY `node_type` (`type`(4)),
  KEY `uid` (`uid`),
  KEY `tnid` (`tnid`),
  KEY `translate` (`translate`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=68574 ;

--
-- Dumping data for table `node`
--

INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(438, 441, 'article', 'en', 'Metaplastic Breast Carcinomas: A Report of Six Cases', 1, 1, 1342756068, 1349958278, 0, 0, 0, 0, 0, 0),
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(442, 445, 'article', 'en', 'Risk Determinants of Peripheral Neuropathy in Patients with Type II Diabetes Mellitus Attending Follow-Up Clinics at University Kebangsaan Malaysia Medical Center (UKMMC): A Cross Sectional Study', 1, 1, 1342756507, 1349967515, 0, 0, 0, 0, 0, 0),
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(1080, 1087, 'conference_paper', 'en', 'AUTOMATED EXTERNAL DEFIBRILLATOR (AED) USE AMONG PARAMEDICS IN THE EMERGENCY DEPARTMENT – WHAT ARE THE OBSTACLES IN USING THE AUTOMATED  EXTERNAL DEFIBRILLATOR IN THE PRE-HOSPITAL CARE SETTINGS ?', 101, 1, 1399278396, 1402561934, 0, 0, 0, 0, 0, 0),
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(1087, 1095, 'conference_paper', 'en', 'Impact of Total Contact Plaster Boot via off loading practice In Trophic Ulcers of the Feet: Case series.', 236, 1, 1402648465, 1402648465, 0, 0, 0, 0, 0, 0),
(1088, 1096, 'issue', 'en', 'Vol. 8 No. 2 : 2013', 64, 1, 1404888220, 1405043193, 0, 0, 0, 0, 0, 0),
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(7380, 7389, 'author', 'en', '☖{GETHACK✲} 24-06-2016 :: Clash Royale cheats real ', 1771, 1, 1466772225, 1466772225, 0, 0, 0, 0, 0, 0),
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(7386, 7395, 'author', 'en', '◟$Hackpack 24/06/2016 :: Star Wars Galactic Defense chea♈t☸s♚ that aCt♒ually work Gems | Android/iOS', 1771, 1, 1466772246, 1466772246, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(7438, 7447, 'author', 'en', '♋{hackgenerator} 24:06:2016 :: Marvel Pinball Hack 2016 hack Go.ld | 2016', 1771, 1, 1466772385, 1466772385, 0, 0, 0, 0, 0, 0),
(7439, 7448, 'author', 'en', '✲(*(❷❷❷)*) :: Subway Surfers cheats for android Coins, Keys | Android/iOS', 1771, 1, 1466772390, 1466772390, 0, 0, 0, 0, 0, 0),
(7440, 7449, 'author', 'en', '☪[~[GETHACK]~] 24-06-2016 :: Clash of Clans Cheats hack Gems', 1771, 1, 1466772395, 1466772395, 0, 0, 0, 0, 0, 0),
(7441, 7450, 'author', 'en', '☓{❽❽❽}☓ :: BLEACH Brave Souls cheats download hack Spirit Orbs | iOS-Android', 1771, 1, 1466772400, 1466772400, 0, 0, 0, 0, 0, 0),
(7442, 7451, 'author', 'en', '▾{*{❽❽�☏�}*} :: Clash Royale c^Heats for android free Gold', 1771, 1, 1466772401, 1466772401, 0, 0, 0, 0, 0, 0),
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(7444, 7453, 'author', 'en', '✮(*(MOD)*) 24:06:2016 ::, MARVEL Avengers Academy cheat download free Shards | Android/iOS', 1771, 1, 1466772405, 1466772405, 0, 0, 0, 0, 0, 0),
(7445, 7454, 'author', 'en', '▼cheat▼ 24/06/2016 :: no survey hack google play Dragon City  Free Food, Gold | 2016', 1771, 1, 1466772407, 1466772407, 0, 0, 0, 0, 0, 0),
(7446, 7455, 'author', 'en', '♿(~(❾❾�:��)~)♿ :: Star Wars Pinball 3 cheat real free Diamond | ANdroid', 1771, 1, 1466772412, 1466772412, 0, 0, 0, 0, 0, 0),
(7447, 7456, 'author', 'en', '♙|¯\\_megahac☇k_/¯|♙ 24/06/2016 :: Candy Crush Soda Saga Hack Tool APK IPA Gold, Level', 1771, 1, 1466772413, 1466772413, 0, 0, 0, 0, 0, 0),
(7448, 7457, 'author', 'en', '✮[*[❹❹❹]*] :: fre*e game hack tools Angry Birds 2 ', 1771, 1, 1466772414, 1466772414, 0, 0, 0, 0, 0, 0),
(7449, 7458, 'author', 'en', '☜[~�;��◢❼❼~] :: NBA LIVE Mobile hack cheats frE◳e Cash', 1771, 1, 1466772414, 1466772414, 0, 0, 0, 0, 0, 0),
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(7453, 7462, 'author', 'en', '☌[~[❹❹❹]~]☌ :: how to hack Clash Royale ', 1771, 1, 1466772430, 1466772430, 0, 0, 0, 0, 0, 0),
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(7455, 7464, 'author', 'en', '❀{~{❶❶❶}~} :: The Walking Dead Road to Survival hack iphone hack Coins, Material', 1771, 1, 1466772431, 1466772431, 0, 0, 0, 0, 0, 0),
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(7463, 7472, 'author', 'en', '◜[*hackpac|k*]◝ :: Mobile Strike cheats android generate Go^ld, Stone | Android', 1771, 1, 1466772467, 1466772467, 0, 0, 0, 0, 0, 0),
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(7472, 7481, 'author', 'en', '❔|¯\\_❸❸❸_/¯|❕ :: Dragon Land Triche free Coins | iOS-Android', 1771, 1, 1466772498, 1466772498, 0, 0, 0, 0, 0, 0),
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(7482, 7491, 'author', 'en', '◎[![mod]!] 24.06.2016 :: Clash Royale cheats hac☡ks free Go.ld, Gems', 1771, 1, 1466772528, 1466772528, 0, 0, 0, 0, 0, 0),
(7483, 7492, 'author', 'en', '▦[!❻❻❻!] :: KENDALL and KYLIE tips cheat◖s hack Gems | iOS-Android', 1771, 1, 1466772528, 1466772528, 0, 0, 0, 0, 0, 0),
(7484, 7493, 'author', 'en', '☊(!(❶❶❶)!)☋ :: Basketball Stars pirater telecharg.er free C.a.s.h. | 2016', 1771, 1, 1466772528, 1466772528, 0, 0, 0, 0, 0, 0),
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(7486, 7495, 'author', 'en', '☧(~(❸❸�☕��)~) :: Digimon Heroes hack telecharger hack Coins | 2016', 1771, 1, 1466772532, 1466772532, 0, 0, 0, 0, 0, 0),
(7487, 7496, 'author', 'en', '☬{!{06/24/2016}!} :: Pet Rescue Saga cheat sheet h☰ack Coins | Android/iOS', 1771, 1, 1466772546, 1466772546, 0, 0, 0, 0, 0, 0),
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(7702, 7711, 'author', 'en', '✺[[FUllhack]]✺ 2.4.06.2016 :: Geometry Dash Triche Gratuit free Gold', 1771, 1, 1466773257, 1466773257, 0, 0, 0, 0, 0, 0),
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(7704, 7713, 'author', 'en', '◕#❶❶❶ :: Killer Zombie cheaTs hacking Gems |♊ Android', 1771, 1, 1466773258, 1466773258, 0, 0, 0, 0, 0, 0),
(7705, 7714, 'author', 'en', '▯[![❷❷❷]!] :: Angry Birds Space Premium cheats iphone ', 1771, 1, 1466773265, 1466773265, 0, 0, 0, 0, 0, 0),
(7706, 7715, 'author', 'en', '♮{!haCkpack!}♮ 24:06:2016 :: Modern Combat 5 Blackout cheats that actu▤ally wor◠k hack Bluecredits, Goldcredits | iOS-A.Ndr!◕oid', 1771, 1, 1466773267, 1466773267, 0, 0, 0, 0, 0, 0),
(7707, 7716, 'author', 'en', '☄[*CHEATS*]☇ 24/06/2016 :: Ninja Saga cheats that work generate Gold, T◣ok♷en', 1771, 1, 1466773276, 1466773276, 0, 0, 0, 0, 0, 0),
(7708, 7717, 'author', 'en', '◣(~(❹❹❹)~) :: Farming Simulator Juwelen| Hack free Money | 2016', 1771, 1, 1466773277, 1466773277, 0, 0, 0, 0, 0, 0),
(7709, 7718, 'author', 'en', '◨{~{❾❾❾}~} :: Clash of Clans cheats apk hack Gems, Gold', 1771, 1, 1466773277, 1466773277, 0, 0, 0, 0, 0, 0),
(7710, 7719, 'author', 'en', '◥|¯\\_�❎��❸❸_/¯|◦ :: Evolution Battle for Utopia tips tr☞icks hack Gems | iOS-Android', 1771, 1, 1466773280, 1466773280, 0, 0, 0, 0, 0, 0),
(7711, 7720, 'author', 'en', '(!(❻❻❻)!) :: KENDALL and KYLIE apk mega mod ', 1771, 1, 1466773280, 1466773280, 0, 0, 0, 0, 0, 0),
(7712, 7721, 'author', 'en', '♇{!{CHEAT}!}♈ 24◙:06:2016 :: Digimon Heroes hack for ios free Coins | Android', 1771, 1, 1466773283, 1466773283, 0, 0, 0, 0, 0, 0),
(7713, 7722, 'author', 'en', '◩{{❾❾❾}}◩ :: NBA LIVE Mobile Hack iPhone iPad Android generate Cas✭h | 2016', 1771, 1, 1466773291, 1466773291, 0, 0, 0, 0, 0, 0),
(7714, 7723, 'author', 'en', '❁[!❸❸❸!] :: Clash Royale on google play downl◈oad  ', 1771, 1, 1466773297, 1466773297, 0, 0, 0, 0, 0, 0),
(7715, 7724, 'author', 'en', '☠|¯\\_�☻��❸❸_/¯| :: Fishing Hook hack ifunbox Hack Money | Androi◨d', 1771, 1, 1466773299, 1466773299, 0, 0, 0, 0, 0, 0),
(7716, 7725, 'author', 'en', '♅(!(H..A..C..K..C..H..E..A..T..S..)!) 24/06/2016 :: Gangstar Vegas cheats online free Key | 2.016', 1771, 1, 1466773300, 1466773300, 0, 0, 0, 0, 0, 0),
(7717, 7726, 'author', 'en', '◿(~❾❾❾~) :: Boom Beach hAck generator ', 1771, 1, 1466773308, 1466773308, 0, 0, 0, 0, 0, 0),
(7718, 7727, 'author', 'en', '❓((H✾ACKED)) 24-06-2016 :: Magic Rush tricheurs f◟ree Gold, G/◰ems | 2016', 1771, 1, 1466773314, 1466773314, 0, 0, 0, 0, 0, 0),
(7719, 7728, 'author', 'en', '♇[!MEGAHACK!]♈ 24-06-2016 :: KENDALL and KYLIE hack for ios ', 1771, 1, 1466773315, 1466773315, 0, 0, 0, 0, 0, 0),
(7720, 7729, 'author', 'en', '▾[![HACKGENERATOR]!]▿ 24:06:2016 :: Heroes Reborn Hack No Survey hack Diamonds; | Android/iOS', 1771, 1, 1466773315, 1466773315, 0, 0, 0, 0, 0, 0),
(7721, 7730, 'author', 'en', '☤{❷❷❷} :: Monkey Boxing cheats for AndRoid free Gold | iOS-Android', 1771, 1, 1466773315, 1466773315, 0, 0, 0, 0, 0, 0),
(7722, 7731, 'author', 'en', '♖(!❺❺❺!) :: Clash Royale Hack frEe Gold | 2016', 1771, 1, 1466773319, 1466773319, 0, 0, 0, 0, 0, 0),
(7723, 7732, 'author', 'en', '◜(~(❶❶❶)~)◜ :: Telecharger This War Of Mine Hack Gratuit hack Ammo, D▭iamond | 2016', 1771, 1, 1466773329, 1466773329, 0, 0, 0, 0, 0, 0),
(7724, 7733, 'author', 'en', '◁~!~M,A,Y,◁ 24:06:2016 :: Real Football 2013 hack safe ', 1771, 1, 1466773334, 1466773334, 0, 0, 0, 0, 0, 0),
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(7726, 7735, 'author', 'en', '☌{*{❼❼❼}*} :: Stick Squad Sniper Contracts Pirater ', 1771, 1, 1466773335, 1466773335, 0, 0, 0, 0, 0, 0),
(7727, 7736, 'author', 'en', '♇¯\\_❷❷❷_/¯♇ :: NBA LIVE Mobile cheat eNgIne generate Cash | iOS-An◻drOid', 1771, 1, 1466773355, 1466773355, 0, 0, 0, 0, 0, 0),
(7728, 7737, 'author', 'en', '✹�.��❹❹ :: MARVEL Avengers Academy cheats iphone. Credits | Android/iOS', 1771, 1, 1466773361, 1466773361, 0, 0, 0, 0, 0, 0),
(7729, 7738, 'author', 'en', '©❹❹❹© :: Adventures Of Mana hack no survey  ', 1771, 1, 1466773366, 1466773366, 0, 0, 0, 0, 0, 0),
(7730, 7739, 'author', 'en', '♪(!(M◞EGAHACK)!)♪ 24.06.2016 :: Clash Royale cheats and hacks Gems', 1771, 1, 1466773366, 1466773366, 0, 0, 0, 0, 0, 0),
(7731, 7740, 'author', 'en', '❄GETHACK 24/06/2016 :: The Walking Dead Road to Survival hack for ios generate Coins, Material | iOS-Android', 1771, 1, 1466773366, 1466773366, 0, 0, 0, 0, 0, 0),
(7732, 7741, 'author', 'en', '▰[!engine!] 24/06/2016 :: Moto Racing Gp 2015 cheats online Cash, Gold | Android', 1771, 1, 1466773367, 1466773367, 0, 0, 0, 0, 0, 0),
(7733, 7742, 'author', 'en', '✿{GENERATOR}✿ 24/06/2016 :: Battlefront Heroes hack tool download hack Food | Android/iOS', 1771, 1, 1466773370, 1466773370, 0, 0, 0, 0, 0, 0),
(7734, 7743, 'author', 'en', '♝[HACKPACK] 24/06/2016 :: GTA 5 Hac❀k free Money', 1771, 1, 1466773371, 1466773371, 0, 0, 0, 0, 0, 0),
(7735, 7744, 'author', 'en', '✴|_/¯❷❷❷¯\\_|✵ :: Clash Royale cheats o◬nline Gold | A!n!d!r!o!i!d!/!i!O!S!', 1771, 1, 1466773373, 1466773373, 0, 0, 0, 0, 0, 0),
(7736, 7745, 'author', 'en', '✴[~[❷❷❷]~] :: Head Soccer Cheat a,pp hack Gold', 1771, 1, 1466773378, 1466773378, 0, 0, 0, 0, 0, 0),
(7737, 7746, 'author', 'en', '✗❼❼❼ :: Kingdoms Lords iPa hack ', 1771, 1, 1466773382, 1466773382, 0, 0, 0, 0, 0, 0),
(7738, 7747, 'author', 'en', '☦[~❶❶❶~]☧ ::☤ h◽ow to hack Ninja Saga Token', 1771, 1, 1466773388, 1466773388, 0, 0, 0, 0, 0, 0),
(7739, 7748, 'author', 'en', '♠[![❽❽❽]!]♣ :: Fishing Hook Hack No Survey ', 1771, 1, 1466773389, 1466773389, 0, 0, 0, 0, 0, 0),
(7740, 7749, 'author', 'en', '❐[!❶❶❶!] :: Dawn Of The Sniper hack for ios ', 1771, 1, 1466773393, 1466773393, 0, 0, 0, 0, 0, 0),
(7741, 7750, 'author', 'en', '☪[~❺❺❺~]☫ :: MARVEL Avengers Academy Glitch hack Cred❂its, Shards', 1771, 1, 1466773398, 1466773398, 0, 0, 0, 0, 0, 0),
(7742, 7751, 'author', 'en', '♎(!ge◣nerator!)♏ 24/06/2016 :: Global Strike c☎heats real ', 1771, 1, 1466773398, 1466773398, 0, 0, 0, 0, 0, 0),
(7743, 7752, 'author', 'en', '✨{~generator~}✫ 24-;06-2016◘ :: Neon Shadow hack software Ammo | 2016', 1771, 1, 1466773400, 1466773400, 0, 0, 0, 0, 0, 0),
(7744, 7753, 'author', 'en', '✒!hackpAck✒ 24/06/2016✖ :: Kingdoms Lords cheats for mobile free Flag, Gems | Android/iOS', 1771, 1, 1466773408, 1466773408, 0, 0, 0, 0, 0, 0),
(7745, 7754, 'author', 'en', '▯_/¯❺❺❺¯\\_▰ :: Domination hack tha◵t works Crown, Gold | Android/iOS', 1771, 1, 1466773410, 1466773410, 0, 0, 0, 0, 0, 0),
(7746, 7755, 'author', 'en', '☉((hacked)) 24:06:2016 :: Clash Royale Hack 2016 ', 1771, 1, 1466773416, 1466773416, 0, 0, 0, 0, 0, 0),
(7747, 7756, 'author', 'en', '☏_/¯HACKPACK¯\\_ 24-06-2016 :: Respawnables hack mod ', 1771, 1, 1466773417, 1466773417, 0, 0, 0, 0, 0, 0),
(7748, 7757, 'author', 'en', '♠❶❶❶♠ :: Basketball Stars cheats apk gEnerate Cash | Android', 1771, 1, 1466773421, 1466773421, 0, 0, 0, 0, 0, 0),
(7749, 7758, 'author', 'en', '❅❾❾❾❅ :: Grand Theft Auto Vice City hack android hack Rp | Android', 1771, 1, 1466773425, 1466773425, 0, 0, 0, 0, 0, 0),
(7750, 7759, 'author', 'en', '✱|¯\\_mod_/¯|✱ 24-06-▢2016. :: Top Eleven 2015 Hack Cheat Juwelen Deutsch! ', 1771, 1, 1466773425, 1466773425, 0, 0, 0, 0, 0, 0),
(7751, 7760, 'author', 'en', '▽~!~❷�❀�❷ :: how to get hack tool Grand Theft Auto Vice City ', 1771, 1, 1466773425, 1466773425, 0, 0, 0, 0, 0, 0),
(7752, 7761, 'author', 'en', '❈#�.��❸❸❈ :: Code de Triche Mobile Strike ', 1771, 1, 1466773426, 1466773426, 0, 0, 0, 0, 0, 0),
(7753, 7762, 'author', 'en', '❝~!~❽❽❽❞ :: Vector Hack Cheats✻ Tra:iner free Coins, Star | iOS-Android', 1771, 1, 1466773432, 1466773432, 0, 0, 0, 0, 0, 0),
(7754, 7763, 'author', 'en', '☽[[❹❹❹]] :: Mobile Strike ha♁ck apk Stone | 2016', 1771, 1, 1466773439, 1466773439, 0, 0, 0, 0, 0, 0),
(7755, 7764, 'author', 'en', '❏(~(❺❺❺)~) :: Boom Beach ipa hack free Diamonds | iOS-Android', 1771, 1, 1466773441, 1466773441, 0, 0, 0, 0, 0, 0),
(7756, 7765, 'author', 'en', '❁❹❹❹❁ :: Hills Of Glory Tip♫s tricks free Money | 2016', 1771, 1, 1466773445, 1466773445, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(7757, 7766, 'author', 'en', '☍[~HACK~]☍ 24/06/2016 :: First Touch Soccer 2015 Online HAck ', 1771, 1, 1466773453, 1466773453, 0, 0, 0, 0, 0, 0),
(7758, 7767, 'author', 'en', '♵¯\\_HAC.KPACK_/¯♵ 24/06/2016 :: Clash Royale hack andro.id no rOot free Gems | 2016', 1771, 1, 1466773457, 1466773457, 0, 0, 0, 0, 0, 0),
(7759, 7768, 'author', 'en', '☬###Hackgene◒rator :: Punch Boxing 3d cheats hacking hack Energy | Android', 1771, 1, 1466773460, 1466773460, 0, 0, 0, 0, 0, 0),
(7760, 7769, 'author', 'en', '✯(!(❼❼❼)!)✯ :: Heroes Charge tips for noobs generAte Gems, Gold', 1771, 1, 1466773460, 1466773460, 0, 0, 0, 0, 0, 0),
(7761, 7770, 'author', 'en', '◶(*(hacked)*) 24/06/2016 :: Lords Mobile hack for ios generate GemS | iOS-Andr.Oid', 1771, 1, 1466773461, 1466773461, 0, 0, 0, 0, 0, 0),
(7762, 7771, 'author', 'en', '✽{~{❶❶❶}~}✽ :: Block Force hack real ', 1771, 1, 1466773462, 1466773462, 0, 0, 0, 0, 0, 0),
(7763, 7772, 'author', 'en', '♅!hackcheat.s 24.✬06.2016 :: Geometry Dash HacK iPhone iPad Android Gold, Level | iOS-..Android', 1771, 1, 1466773464, 1466773464, 0, 0, 0, 0, 0, 0),
(7764, 7773, 'author', 'en', '☤(hackengine)☤ 24:0☊6:2016 :: Samurai Shodown II Hack To♵ol AP♒K IPA ', 1771, 1, 1466773467, 1466773467, 0, 0, 0, 0, 0, 0),
(7765, 7774, 'author', 'en', '✤(*(GENERATOR)*)✤ 24-06-2016 ::^ Clash Royale hack android no root hack Gold | Android/iOS', 1771, 1, 1466773469, 1466773469, 0, 0, 0, 0, 0, 0),
(7766, 7775, 'author', 'en', '★❹❹❹★ :: Geometry Dash Hacken free Gold | iOS-❜Android', 1771, 1, 1466773482, 1466773482, 0, 0, 0, 0, 0, 0),
(7767, 7776, 'author', 'en', '☈FULLHACK☈ 24:06:2016 :: Sandstorm Pirate Wars hack mod generate Power Cells', 1771, 1, 1466773484, 1466773484, 0, 0, 0, 0, 0, 0),
(7768, 7777, 'author', 'en', '♧{❶❶❶}♨ :: Kritika hack real Health | Android/iOS', 1771, 1, 1466773489, 1466773489, 0, 0, 0, 0, 0, 0),
(7769, 7778, 'author', 'en', '◬¯\\_❸❸❸_/¯ :: Heroes Dragon Age Hack Tool APK IPA Gems | Android/iOS', 1771, 1, 1466773495, 1466773495, 0, 0, 0, 0, 0, 0),
(7770, 7779, 'author', 'en', '♊[24.06.2016] :: Weed Firm 2 ch.eat that works geNerate Cash | Android', 1771, 1, 1466773500, 1466773500, 0, 0, 0, 0, 0, 0),
(7771, 7780, 'author', 'en', '▷{!hacked!} 24.06.2016 :: BLEACH Brave Souls ha☣ck proof hack Spirit Orbs | iOS-Android', 1771, 1, 1466773501, 1466773501, 0, 0, 0, 0, 0, 0),
(7772, 7781, 'author', 'en', '❄[![megahack]!]❄ 24:06:2016 :: Star Wars Galaxy of Heroes APK IPA Mod ', 1771, 1, 1466773504, 1466773504, 0, 0, 0, 0, 0, 0),
(7773, 7782, 'author', 'en', '☲(!(❾❾❾)!)☲ :: Fishing Hook cheat code entry free Gold, Money | iOS-Android', 1771, 1, 1466773504, 1466773504, 0, 0, 0, 0, 0, 0),
(7774, 7783, 'author', 'en', '◨{!MAY!}◩ :: Clash Royale Hack No Survey free Gems', 1771, 1, 1466773504, 1466773504, 0, 0, 0, 0, 0, 0),
(7775, 7784, 'author', 'en', '☶[❻❻�®��]☶ :: cheats for Bubble Witch 2 Saga Coins, Gold | iOS-Android', 1771, 1, 1466773520, 1466773520, 0, 0, 0, 0, 0, 0),
(7776, 7785, 'author', 'en', '◍(!hackt❇ool!)◎ ::♤ Mutans H.ack No Survey free♟ Co.i♖ns, Gold | iOS-A◅nDroid', 1771, 1, 1466773521, 1466773521, 0, 0, 0, 0, 0, 0),
(7777, 7786, 'author', 'en', '▴[*❹❹❹*] :: Shooting Showdown hack no jaibrea♭k hack Diamond | 2016', 1771, 1, 1466773521, 1466773521, 0, 0, 0, 0, 0, 0),
(7778, 7787, 'author', 'en', '☙[![❾❾❾]!]☙ :: Global Strike hack apk generat◰e Gold, GP | 2016', 1771, 1, 1466773522, 1466773522, 0, 0, 0, 0, 0, 0),
(7779, 7788, 'author', 'en', '♰{!❷❷❷!}♰ :: Angry Birds Rio Triche hack Slingscope, Superseed | Android', 1771, 1, 1466773531, 1466773531, 0, 0, 0, 0, 0, 0),
(7780, 7789, 'author', 'en', '♣[2016] 24/06/2016 :: Fishing Mania 3d hack tool hack Energy, Gold | Android', 1771, 1, 1466773534, 1466773534, 0, 0, 0, 0, 0, 0),
(7781, 7790, 'author', 'en', '☶¯\\_hacktool_/¯ 24/❏06/2016 :: Robocide ha♴ck mod apk ', 1771, 1, 1466773537, 1466773537, 0, 0, 0, 0, 0, 0),
(7782, 7791, 'author', 'en', '☿(*(24-06-2016)*)☿ :: Ski Safari cheats for mobile ', 1771, 1, 1466773537, 1466773537, 0, 0, 0, 0, 0, 0),
(7783, 7792, 'author', 'en', '☸(~❹❹❹~)☹ :: Dragon City Glitch generate Food | Android', 1771, 1, 1466773537, 1466773537, 0, 0, 0, 0, 0, 0),
(7784, 7793, 'author', 'en', '✪[![❺❺❺]!]♠ :: Clash of Clans hack proof ', 1771, 1, 1466773548, 1466773548, 0, 0, 0, 0, 0, 0),
(7785, 7794, 'author', 'en', '❉❺❺❺❊ :: Clash Royale Cheats ', 1771, 1, 1466773551, 1466773551, 0, 0, 0, 0, 0, 0),
(7786, 7795, 'author', 'en', '◟[~[❺❺❺]~] :: Pocket Stables cheats hacks free Coins | 2016', 1771, 1, 1466773555, 1466773555, 0, 0, 0, 0, 0, 0),
(7787, 7796, 'author', 'en', '♧❼❼❼♧ :: Clash Royale APK IPA Mod ', 1771, 1, 1466773557, 1466773557, 0, 0, 0, 0, 0, 0),
(7788, 7797, 'author', 'en', '☌###❶❶❶☌ :: Marvel Contest Of Champions cheats a❁n*droid ', 1771, 1, 1466773563, 1466773563, 0, 0, 0, 0, 0, 0),
(7789, 7798, 'author', 'en', '☋|_/¯❾❾.�|��¯\\_|☌ :: Ultimate Robot Fighting Online Generator generate Matchpoint | Android/iOS', 1771, 1, 1466773564, 1466773564, 0, 0, 0, 0, 0, 0),
(7790, 7799, 'author', 'en', '♆{!❷❷❷!}♆ :: KENDALL and KYLIE hacked apk free Gems, Ca.sh | Android', 1771, 1, 1466773566, 1466773566, 0, 0, 0, 0, 0, 0),
(7791, 7800, 'author', 'en', '❕❹❹❹❕ :: cheats for Hay Day Coins', 1771, 1, 1466773567, 1466773567, 0, 0, 0, 0, 0, 0),
(7792, 7801, 'author', 'en', '◁[~[�☦��❼❼]~] :: BLEACH Brave Souls Hack No Surve☭y free Spirit Orbs | 2016', 1771, 1, 1466773571, 1466773571, 0, 0, 0, 0, 0, 0),
(7793, 7802, 'author', 'en', '✺[[❾❾❾]]✻ :: Clash Royale cheats i\\p\\a\\d\\ free Gems | 2016.', 1771, 1, 1466773574, 1466773574, 0, 0, 0, 0, 0, 0),
(7794, 7803, 'author', 'en', '♥[*cheats*] 24:06:2016 :: Pirate Legends Cheats android ANchor, Toe◽s. | AndroiD', 1771, 1, 1466773579, 1466773579, 0, 0, 0, 0, 0, 0),
(7795, 7804, 'author', 'en', '❜(!GE.THACK!)❝ 24-06-2016 :: Star Wars Pinball 3 hack tool online generate Ball | 2016', 1771, 1, 1466773581, 1466773581, 0, 0, 0, 0, 0, 0),
(7796, 7805, 'author', 'en', '☝[~GENERATOR~]☞ 24-06-2016 :: Mobile Strike p:irater telecharger hack Gold | 2016', 1771, 1, 1466773588, 1466773588, 0, 0, 0, 0, 0, 0),
(7797, 7806, 'author', 'en', '♗_/¯�✾�❺❺¯\\_♘ :: Nba 2k15 hack apk free Diamond | 2016', 1771, 1, 1466773590, 1466773590, 0, 0, 0, 0, 0, 0),
(7798, 7807, 'author', 'en', '▶(!❼❼❼!)▷ :: Real Steel World Robot Boxing mod hack Coins | Android/iOS', 1771, 1, 1466773593, 1466773593, 0, 0, 0, 0, 0, 0),
(7799, 7808, 'author', 'en', '♢(~❾❾❾~) :: how to hac✫k The Sims Free Play hack Simoleons', 1771, 1, 1466773593, 1466773593, 0, 0, 0, 0, 0, 0),
(7800, 7809, 'author', 'en', '✵{!{❷❷.❷}!}✶ :: Dawn Of The Sniper Cheats ', 1771, 1, 1466773596, 1466773596, 0, 0, 0, 0, 0, 0),
(7801, 7810, 'author', 'en', '▾~!~HACK/❒ED▾ 24.06|.2016 :: Clash Royale cheats. tool ', 1771, 1, 1466773597, 1466773597, 0, 0, 0, 0, 0, 0),
(7802, 7811, 'author', 'en', '☃[!❸❸*❸!]☄ :: Farmville 2 cheats reviews ', 1771, 1, 1466773600, 1466773600, 0, 0, 0, 0, 0, 0),
(7803, 7812, 'author', 'en', '♩[~[�✦�☐�❺❺]~]♩ :: Virtual Tennis Challenge cheats online free Money', 1771, 1, 1466773605, 1466773605, 0, 0, 0, 0, 0, 0),
(7804, 7813, 'author', 'en', '❓{!{❼❼❼}!}❓ :: KENDALL and KYLIE hack, apk free Cash | AndroId/iOS', 1771, 1, 1466773609, 1466773609, 0, 0, 0, 0, 0, 0),
(7805, 7814, 'author', 'en', '◪{!{24.06.2016}!}◪ :: Heroes Reborn Hack iphone generate Coins', 1771, 1, 1466773611, 1466773611, 0, 0, 0, 0, 0, 0),
(7806, 7815, 'author', 'en', '✽❻❻✩❻✾ :: NBA LIVE Mobile Tips tricks Coi/ns,☣ Cash | Android/iOS', 1771, 1, 1466773615, 1466773615, 0, 0, 0, 0, 0, 0),
(7807, 7816, 'author', 'en', '☭$cheats☮ :: Ace Fishing hack, no root generate Energy | 2016', 1771, 1, 1466773616, 1466773616, 0, 0, 0, 0, 0, 0),
(7808, 7817, 'author', 'en', '☎(!(online)!) 24:06:2016 :: Plants Zombie 2 c.h.e.a.t. app hack Key | Android', 1771, 1, 1466773618, 1466773618, 0, 0, 0, 0, 0, 0),
(7809, 7818, 'author', 'en', '♊[!❾❾.❾!]♊ :: Clash Royale cheats ios ', 1771, 1, 1466773621, 1466773621, 0, 0, 0, 0, 0, 0),
(7810, 7819, 'author', 'en', '✫{*❻❻❻*}✫ :: Grand Theft Auto China Town ch✺eats tool Money | 2016', 1771, 1, 1466773626, 1466773626, 0, 0, 0, 0, 0, 0),
(7811, 7820, 'author', 'en', '☶[*[❶❶❶]*] :: Smashy City M.od Apk ', 1771, 1, 1466773633, 1466773633, 0, 0, 0, 0, 0, 0),
(7812, 7821, 'author', 'en', '◧|¯\\_MAY_/¯|◧ 24.06.2016 ::, Sandstorm Pirate Wars hack android no root hack Power CeLls, Bolts | Andr✖oid/iOS', 1771, 1, 1466773635, 1466773635, 0, 0, 0, 0, 0, 0),
(7813, 7822, 'author', 'en', '♡(!❹❹❹!)♡ :: The Sims Free Play tips cheats ', 1771, 1, 1466773637, 1466773637, 0, 0, 0, 0, 0, 0),
(7814, 7823, 'author', 'en', '▰|_/¯hackgenerat☋or¯\\_|▰ 24/06/2016 :: Dragon Land hack no jaibreak ', 1771, 1, 1466773639, 1466773639, 0, 0, 0, 0, 0, 0),
(7815, 7824, 'author', 'en', '◄{~{❹❹❹}~}◅ :: Gangstar Vegas hack cheats Key, Gems | 2016', 1771, 1, 1466773640, 1466773640, 0, 0, 0, 0, 0, 0),
(7816, 7825, 'author', 'en', '☈{~❷❷�*�~}☈ :: Fruit Ninja Hack generate Fruit, Juic.e | Android', 1771, 1, 1466773645, 1466773645, 0, 0, 0, 0, 0, 0),
(7817, 7826, 'author', 'en', '❈[![❹❹❹]!] :: Paradise Bay cheats for mobile ', 1771, 1, 1466773650, 1466773650, 0, 0, 0, 0, 0, 0),
(7818, 7827, 'author', 'en', '✣{*{2016}*}✣ :: Pocket Stables hack cod☧es free Bucks | iOS-Android', 1771, 1, 1466773655, 1466773655, 0, 0, 0, 0, 0, 0),
(7819, 7828, 'author', 'en', '✶((❸❸❸))✷ :: World of Tanks Online GeNerator ', 1771, 1, 1466773664, 1466773664, 0, 0, 0, 0, 0, 0),
(7820, 7829, 'author', 'en', '△(~(❹❹❹)~) :: Hay Day c♊heats codes hack Coins, Diam▿ond◅s | Android', 1771, 1, 1466773667, 1466773667, 0, 0, 0, 0, 0, 0),
(7821, 7830, 'author', 'en', '☡|¯\\_�◉�❻❻_/¯|☢ :: Clash Royale Online Hack ', 1771, 1, 1466773668, 1466773668, 0, 0, 0, 0, 0, 0),
(7822, 7831, 'author', 'en', '▮[![❹❹❹]!]▯ :: no survey hack google play MARVEL Avengers Academy  free Shards | Andro☶id', 1771, 1, 1466773670, 1466773670, 0, 0, 0, 0, 0, 0),
(7823, 7832, 'author', 'en', '❊[*[ENGINE]*]❊ 24:06:2016 :: Clash of Clans hack tool generatoR ', 1771, 1, 1466773680, 1466773680, 0, 0, 0, 0, 0, 0),
(7824, 7833, 'author', 'en', '❅{!{HACKPACK}!}❆ 24/06/2016 :: Dragon Mania Legends c;h;e;a;t; app hack Gems, Gold | Android', 1771, 1, 1466773684, 1466773684, 0, 0, 0, 0, 0, 0),
(7825, 7834, 'author', 'en', '✡{*❺❺❺*}✡ :: Tanki Online hack codes ', 1771, 1, 1466773686, 1466773686, 0, 0, 0, 0, 0, 0),
(7826, 7835, 'author', 'en', '☯[![CHEATS]!]☯ :: Bubble Shooter cheats for iphone hack EnErgy | Android/iOS', 1771, 1, 1466773686, 1466773686, 0, 0, 0, 0, 0, 0),
(7827, 7836, 'author', 'en', '♶[~❷�.�.�❷\\~]♷ :: Star Wars Galactic Defense hack no downl❐o.aD  ', 1771, 1, 1466773688, 1466773688, 0, 0, 0, 0, 0, 0),
(7828, 7837, 'author', 'en', '❗[[hackTool]]❗ :: Clash Royale apk meg/a mod generate Go.ld,^ Gems', 1771, 1, 1466773691, 1466773691, 0, 0, 0, 0, 0, 0),
(7829, 7838, 'author', 'en', '♭(!CHEAT!) 24:|06:2016 :: Legion Of Heroes hack codes ', 1771, 1, 1466773702, 1466773702, 0, 0, 0, 0, 0, 0),
(7830, 7839, 'author', 'en', '☳_/¯❶❶❶¯\\_ :: Pet Rescue Saga cheat apk ', 1771, 1, 1466773705, 1466773705, 0, 0, 0, 0, 0, 0),
(7831, 7840, 'author', 'en', '✬~!~❾❾❾ :: Angry Birds Space Premium cheat engin:e ', 1771, 1, 1466773707, 1466773707, 0, 0, 0, 0, 0, 0),
(7832, 7841, 'author', 'en', '◅#❺�♃��❺◅ :: Front Line Commando hack genera▿tor ', 1771, 1, 1466773717, 1466773717, 0, 0, 0, 0, 0, 0),
(7833, 7842, 'author', 'en', '❗{~{❼❼❼}~}❗ :: BLEACH Brave Souls hac♍k wo✵rk ', 1771, 1, 1466773718, 1466773718, 0, 0, 0, 0, 0, 0),
(7834, 7843, 'author', 'en', '♊{~❺❺❺~} :: Cars Fast As Lightning hack aNdroid hack CoinS | Android', 1771, 1, 1466773720, 1466773720, 0, 0, 0, 0, 0, 0),
(7835, 7844, 'author', 'en', '♷!❽❽❽♷ :: Future Fight cheat apk ', 1771, 1, 1466773720, 1466773720, 0, 0, 0, 0, 0, 0),
(7836, 7845, 'author', 'en', '◫❾�❁��❾◬ :|: The Simpsons Tapped Out Triche generat▯e Coins, Donuts | iOS-Andr▿oid', 1771, 1, 1466773722, 1466773722, 0, 0, 0, 0, 0, 0),
(7837, 7846, 'author', 'en', '◚(~(❺\\❺❺)~)◚ :.:. Fast Furious Legacy Hack ', 1771, 1, 1466773728, 1466773728, 0, 0, 0, 0, 0, 0),
(7838, 7847, 'author', 'en', '✿{!❻❻❻!}✿ :: Hack Para Ski Safari CoiNs | 2.0.1.6.', 1771, 1, 1466773737, 1466773737, 0, 0, 0, 0, 0, 0),
(7839, 7848, 'author', 'en', '▲|¯\\_❹❹❹_/¯| :: Boom Beach Chea❁ts Wood, Diamonds | 2016', 1771, 1, 1466773739, 1466773739, 0, 0, 0, 0, 0, 0),
(7840, 7849, 'author', 'en', '☍(~(❶�!�❶)~) :: Clash Royale cheats ios ', 1771, 1, 1466773743, 1466773743, 0, 0, 0, 0, 0, 0),
(7841, 7850, 'author', 'en', '✬{~H.A.C.K.C.H.E.A.T.S.~}✭ 24:06:2016 :: Evolution Battle for Utopia Glitc☬h aN❁d Glit/ches generate Di.amonds | Android', 1771, 1, 1466773747, 1466773747, 0, 0, 0, 0, 0, 0),
(7842, 7851, 'author', 'en', '◼(!❾❾❾!) :: Terraria Triche GRatuit free Coins | 2016❕', 1771, 1, 1466773750, 1466773750, 0, 0, 0, 0, 0, 0),
(7843, 7852, 'author', 'en', '◙(*(❼❼❼)*)◚ :◻: Taichi Panda cheats hacking ', 1771, 1, 1466773752, 1466773752, 0, 0, 0, 0, 0, 0),
(7844, 7853, 'author', 'en', '☽{!{ENGINE}!}☽ :: Plague hack for ip♦hone ', 1771, 1, 1466775751, 1466775751, 0, 0, 0, 0, 0, 0),
(7845, 7854, 'author', 'en', '✼[!❾❾❾!]✽ :: Sniper Swat Fps hack no download  ', 1771, 1, 1466775752, 1466775752, 0, 0, 0, 0, 0, 0),
(7846, 7855, 'author', 'en', '◒(*hacktool*)◓ 24-06-2016 ::◉ Clash Royale Hack Deutsch ', 1771, 1, 1466775754, 1466775754, 0, 0, 0, 0, 0, 0),
(7847, 7856, 'author', 'en', '❊(~❼❼❼~)❋ :: Darkness Reborn apk cheats ', 1771, 1, 1466775756, 1466775756, 0, 0, 0, 0, 0, 0),
(7848, 7857, 'author', 'en', '▿❼❼�▣�� :: Clash Of King Mod Apk Food |☗ Android/iOS', 1771, 1, 1466775757, 1466775757, 0, 0, 0, 0, 0, 0),
(7849, 7858, 'author', 'en', '☳[❸❸❸]☴ :: Astuces Pour Contract Killer Sniper ', 1771, 1, 1466775775, 1466775775, 0, 0, 0, 0, 0, 0),
(7850, 7859, 'author', 'en', '♔❶❶❶ :: Bubble Shooter hack t♑ool download generate Diamond | Android/iOS', 1771, 1, 1466775776, 1466775776, 0, 0, 0, 0, 0, 0),
(7851, 7860, 'author', 'en', '®{!{❹❹❹}!}☯ :: Angry Birds 2 hack no survey  ', 1771, 1, 1466775776, 1466775776, 0, 0, 0, 0, 0, 0),
(7852, 7861, 'author', 'en', '☵~!~CHEATS☶ 24.06.20/16 :: Code de. Triche Lords Mobile g☑e*nerate Gems | Android/iOS', 1771, 1, 1466775780, 1466775780, 0, 0, 0, 0, 0, 0),
(7853, 7862, 'author', 'en', '✡❹❹❹ :: Clash Royale hack no jailbreak free Gold', 1771, 1, 1466775780, 1466775780, 0, 0, 0, 0, 0, 0),
(7854, 7863, 'author', 'en', '♪((�.�♉�❹❹))♪ :: Street Race Rivals cheats for iphone ', 1771, 1, 1466775799, 1466775799, 0, 0, 0, 0, 0, 0),
(7855, 7864, 'author', 'en', '◞[~❼❼❼~] :: Angry Birds Space Premium cheats apk ', 1771, 1, 1466775800, 1466775800, 0, 0, 0, 0, 0, 0),
(7856, 7865, 'author', 'en', '◉{�♬��❺�.��}◉ :: Clash Royale Hack Cheat Juwelen Deutsch! free Gold, G..e..m..s.. | 2016', 1771, 1, 1466775801, 1466775801, 0, 0, 0, 0, 0, 0),
(7857, 7866, 'author', 'en', '☛[!❶❶❶|!]☛ :: Iron Knights cheats iph.o!ne Gems, Gold | Android', 1771, 1, 1466775805, 1466775805, 0, 0, 0, 0, 0, 0),
(7858, 7867, 'author', 'en', '❙[[❻❻❻]]❙ :: Boboiboy Power Spheres Hack Gra.tis ', 1771, 1, 1466775805, 1466775805, 0, 0, 0, 0, 0, 0),
(7859, 7868, 'author', 'en', '▥{*{cHeat}*} 24:06:2016 ::  free M!a!t!e!r!i!a!l! |/ Android', 1771, 1, 1466775823, 1466775823, 0, 0, 0, 0, 0, 0),
(7860, 7869, 'author', 'en', '▹(*❷❷❷*)► :: Mobile Strike hack ifu.nbox free Gold', 1771, 1, 1466775826, 1466775826, 0, 0, 0, 0, 0, 0),
(7861, 7870, 'author', 'en', '♂[FUL☌LHACK] 24.06.2016 :: Cars Fast As Lightning hack mac. generate Coins, Gems | Android/iOS', 1771, 1, 1466775826, 1466775826, 0, 0, 0, 0, 0, 0),
(7862, 7871, 'author', 'en', '☆2016✯ 24:06:2016 :: Clash of Clans Online Hack ', 1771, 1, 1466775830, 1466775830, 0, 0, 0, 0, 0, 0),
(7863, 7872, 'author', 'en', '△[[❶❶❶]]▴ :: Naughty Kitties hack tool onlin☌E ', 1771, 1, 1466775830, 1466775830, 0, 0, 0, 0, 0, 0),
(7864, 7873, 'author', 'en', '○{*❶❶❶*}○ :: The Walking Dead Road to Survival cheats ha✰cking ', 1771, 1, 1466775844, 1466775844, 0, 0, 0, 0, 0, 0),
(7865, 7874, 'author', 'en', '♥|¯\\_❼❼❼_/¯| :: KENDALL and KYLIE Juwelen Hack Gems, C.ash', 1771, 1, 1466775849, 1466775849, 0, 0, 0, 0, 0, 0),
(7866, 7875, 'author', 'en', '☏{!❷❷❷!}☐ :: Hack Para Clash Royale ', 1771, 1, 1466775849, 1466775849, 0, 0, 0, 0, 0, 0),
(7867, 7876, 'author', 'en', '☇{!❶❶►❶!} :: Pool Billiards Pro hack mod Gold | 2016', 1771, 1, 1466775853, 1466775853, 0, 0, 0, 0, 0, 0),
(7868, 7877, 'author', 'en', '◭{~GENERATO▴R~} 24.06.2016\\◢ :: City Ville cheat engine generate◙ Money | Android/iOS', 1771, 1, 1466775854, 1466775854, 0, 0, 0, 0, 0, 0),
(7869, 7878, 'author', 'en', '◙{{HACKGENERATOR}}◚ 24-06-2016 :: AstucEs Po✓ur Grand Theft Auto III generate Rp | Android/IOS', 1771, 1, 1466775866, 1466775866, 0, 0, 0, 0, 0, 0),
(7870, 7879, 'author', 'en', '☆#fullhack :: Hay Day hack no root ', 1771, 1, 1466775872, 1466775872, 0, 0, 0, 0, 0, 0),
(7871, 7880, 'author', 'en', '❈engine 24/06/2016 :: Clash Royale apk cheats Coins, Gold | 20!16', 1771, 1, 1466775872, 1466775872, 0, 0, 0, 0, 0, 0),
(7872, 7881, 'author', 'en', '✡{{engine}}✢ 24-06-2016 :::: NBA LIVE Mobile cheat engi.ne ', 1771, 1, 1466775875, 1466775875, 0, 0, 0, 0, 0, 0),
(7873, 7882, 'author', 'en', '✩(*hackpack*)✓ 24.06.2016 :: Neon Shadow cheats tha✒t actually work free HEalth | Android', 1771, 1, 1466775876, 1466775876, 0, 0, 0, 0, 0, 0),
(7874, 7883, 'author', 'en', '♗06-24-2♽016♘ :: Hungry Shark Evolution cheat tool ', 1771, 1, 1466775888, 1466775888, 0, 0, 0, 0, 0, 0),
(7875, 7884, 'author', 'en', '✵MAY✵ :: Age Of Sparta Online Hack free Gold, Gems | Android/iOS', 1771, 1, 1466775893, 1466775893, 0, 0, 0, 0, 0, 0),
(7876, 7885, 'author', 'en', '♿{*{❼❼❼}*}♀ :: Clash Royale Hack CheaT Juwelen DEutsch! Gems', 1771, 1, 1466775894, 1466775894, 0, 0, 0, 0, 0, 0),
(7877, 7886, 'author', 'en', '⚓{❷❷❷}﨏 :: Clash Royale cheat that works free Gold, Gems | Android/iOS', 1771, 1, 1466775898, 1466775898, 0, 0, 0, 0, 0, 0),
(7878, 7887, 'author', 'en', '◜[❸❸❸]◝ :.:. Code de TriC:he Fishing Hook free Money | iOS-Android', 1771, 1, 1466775899, 1466775899, 0, 0, 0, 0, 0, 0),
(7879, 7888, 'author', 'en', '❐❹❹�❐�❐ :: Rolling Sky hack safe ', 1771, 1, 1466775910, 1466775910, 0, 0, 0, 0, 0, 0),
(7880, 7889, 'author', 'en', '♲[!❷❷❷!]♲ :: Clear Vision 3 cheat real generate Token | Androi✺d', 1771, 1, 1466775917, 1466775917, 0, 0, 0, 0, 0, 0),
(7881, 7890, 'author', 'en', '♤(!(�..�..�..�..�..�..�..�..�..)!) :: Stick Squad Modern Shooter cheats i..p..h..o..n..e.. fr◑ee D❂iaMond | Android/iOS', 1771, 1, 1466775919, 1466775919, 0, 0, 0, 0, 0, 0),
(7882, 7891, 'author', 'en', '☍[!❼❼❼!] :: Warlings Armageddon Triche ', 1771, 1, 1466775923, 1466775923, 0, 0, 0, 0, 0, 0),
(7883, 7892, 'author', 'en', '✈(*ju♸ne*) 24-06-2016 :: Clash Royale cheats Onli,ne hack Gem▫s', 1771, 1, 1466775923, 1466775923, 0, 0, 0, 0, 0, 0),
(7884, 7893, 'author', 'en', '♾!❷�♆��❷♿ :: MARVEL Avengers Academy hack android CRedits | Android/iOS', 1771, 1, 1466775932, 1466775932, 0, 0, 0, 0, 0, 0),
(7885, 7894, 'author', 'en', '☮###❸❸❸ :: Dungeon Hunter 5 hack no jaibreak ', 1771, 1, 1466775938, 1466775938, 0, 0, 0, 0, 0, 0),
(7886, 7895, 'author', 'en', '☣(~(❺❺❺)~)☤ :: Power Pingpong O☣nli♸ne Hack hack Infinitycoins | A:n:d:r:o:i:d:/:i:O:S:', 1771, 1, 1466775942, 1466775942, 0, 0, 0, 0, 0, 0),
(7887, 7896, 'author', 'en', '☃[~❺❺❺~]☃ :: Evolution Battle for Utopia Tips and Tricks ^', 1771, 1, 1466775944, 1466775944, 0, 0, 0, 0, 0, 0),
(7888, 7897, 'author', 'en', '☈(*❾❾❾*)☈ :: Monster Warlord hack real generate H|e|a|l|t|h| | AnDroid/iOS', 1771, 1, 1466775945, 1466775945, 0, 0, 0, 0, 0, 0),
(7889, 7898, 'author', 'en', '▴[*[❸�|��❸]*] ::  Coins | 2016', 1771, 1, 1466775957, 1466775957, 0, 0, 0, 0, 0, 0),
(7890, 7899, 'author', 'en', '☚[*[❸❸❸]*]☛ :: iSlash Heroes hack android no root ', 1771, 1, 1466775959, 1466775959, 0, 0, 0, 0, 0, 0),
(7891, 7900, 'author', 'en', '☗❽❽❽☗ :: The Sims Free Play hack tool online ', 1771, 1, 1466775964, 1466775964, 0, 0, 0, 0, 0, 0),
(7892, 7901, 'author', 'en', '▧❹❹❹▨ :: Vector hack cheats ', 1771, 1, 1466775967, 1466775967, 0, 0, 0, 0, 0, 0),
(7893, 7902, 'author', 'en', '▰❻❻�.�▱ :: Boom Beach cheats apk ', 1771, 1, 1466775969, 1466775969, 0, 0, 0, 0, 0, 0),
(7894, 7903, 'author', 'en', '♷�.�❻❻♷ :: Clash Royale Hack 2016 generate Gold | 2016', 1771, 1, 1466776005, 1466776005, 0, 0, 0, 0, 0, 0),
(7895, 7904, 'author', 'en', '♉!HACKCHEATS :: Candy Crush Soda Saga pirater telecharger Level | iOS-Android', 1771, 1, 1466776007, 1466776007, 0, 0, 0, 0, 0, 0),
(7896, 7905, 'author', 'en', '✱{~{❼❼❼}~}✲ :: Clash of Clans hack mod apk generate Gems, Gold | Android/iOS', 1771, 1, 1466776009, 1466776009, 0, 0, 0, 0, 0, 0),
(7897, 7906, 'author', 'en', '◭~!~gene♷rator◮ 2♩4/06♴/2016 :: Code de Triche iSlash Heroes ', 1771, 1, 1466776014, 1466776014, 0, 0, 0, 0, 0, 0),
(7898, 7907, 'author', 'en', '☟{~{�●��❹❹}~} :: Lords Mobile Hack Cydia Gold | iOS-Andr◭oid', 1771, 1, 1466776015, 1466776015, 0, 0, 0, 0, 0, 0),
(7899, 7908, 'author', 'en', '♽{!{❼❼❼}!} :: Warlings Armageddon cheats online Gold, Stars', 1771, 1, 1466776026, 1466776026, 0, 0, 0, 0, 0, 0),
(7900, 7909, 'author', 'en', '▴{!hackgenerat;oR!} :|:| Hollywood Story hack mod ', 1771, 1, 1466776029, 1466776029, 0, 0, 0, 0, 0, 0),
(7901, 7910, 'author', 'en', '☶[*[06/24/2016]*] :: Dawn Of The Sniper cheat sheet gener:a✒te Cash | Android/iOS', 1771, 1, 1466776034, 1466776034, 0, 0, 0, 0, 0, 0),
(7902, 7911, 'author', 'en', '♆(~(engine)~)♇ 24-06-2016! :: Heroes Charge cheat❁s download free Gems | 2|016', 1771, 1, 1466776037, 1466776037, 0, 0, 0, 0, 0, 0),
(7903, 7912, 'author', 'en', '♑{!{ONLINE}!} 24:06:2016 :: Clash of Clans hack no jailbreak free Gems | Android/iOS', 1771, 1, 1466776040, 1466776040, 0, 0, 0, 0, 0, 0),
(7904, 7913, 'author', 'en', '♓[![❹❹❹]!]♓ :: Batlle Camp Cheats 2016 free Energy | 2016', 1771, 1, 1466776045, 1466776045, 0, 0, 0, 0, 0, 0),
(7905, 7914, 'author', 'en', '❔{~{�..�..�..�..�..�..�..�..�..}~} :: Clash Royale cheats that work ', 1771, 1, 1466776050, 1466776050, 0, 0, 0, 0, 0, 0),
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(7907, 7916, 'author', 'en', '❆{{fullhack}}❇ :: Candy Crush Saga Mod Apk ', 1771, 1, 1466776057, 1466776057, 0, 0, 0, 0, 0, 0),
(7908, 7917, 'author', 'en', '☼MEGAHACK☼ 24-06-2016 :: MARVEL Avengers Academy cheats online hack Credits, Shards | iOS-Android', 1771, 1, 1466776063, 1466776063, 0, 0, 0, 0, 0, 0),
(7909, 7918, 'author', 'en', '◁|¯\\_FULLHACK_/¯| 24.06.2016 :: Evolution Battle for Utopia cheats that actually w,ork DiAmonds', 1771, 1, 1466776067, 1466776067, 0, 0, 0, 0, 0, 0),
(7910, 7919, 'author', 'en', '▴|_/¯2016¯\\_| :: Plants Zombie 2 ipa hack ', 1771, 1, 1466776070, 1466776070, 0, 0, 0, 0, 0, 0),
(7911, 7920, 'author', 'en', '♕{~h◍ack~}♕ 24:06:2016 :: Dead Trigger 2 hack Cheats free G♺old', 1771, 1, 1466776074, 1466776074, 0, 0, 0, 0, 0, 0),
(7912, 7921, 'author', 'en', '◭(!❼❼�☀�!)◮ :: MARVEL Avengers Academy hack for iPhone generate Credits | Android/iOS', 1771, 1, 1466776079, 1466776079, 0, 0, 0, 0, 0, 0),
(7913, 7922, 'author', 'en', '♓[![cheat]!]♔ 24-06-.2016 :: Air Attack Hd Cheats 2016 generate M|o|n|e|y|', 1771, 1, 1466776085, 1466776085, 0, 0, 0, 0, 0, 0),
(7914, 7923, 'author', 'en', '☇[!❷✧❷❷!]☈ :: Clash Royale hack mod ', 1771, 1, 1466776089, 1466776089, 0, 0, 0, 0, 0, 0),
(7915, 7924, 'author', 'en', '✽(❺❺❺)✾ :: Code de Triche Heroes Dragon Age generate Gems | iOS-A.ndroid', 1771, 1, 1466776092, 1466776092, 0, 0, 0, 0, 0, 0),
(7916, 7925, 'author', 'en', '!hackengine 24:06:2016 :: KENDALL and KYLIE h|a|c|k| ios ', 1771, 1, 1466776097, 1466776097, 0, 0, 0, 0, 0, 0),
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(8028, 8037, 'author', 'en', '❅(*(GENERATOR)*) 24/06/2✖016 :: NBA LIVE Mobile cheats for android CoinS, Cash | iOS-Android', 1771, 1, 1466776647, 1466776647, 0, 0, 0, 0, 0, 0),
(8029, 8038, 'author', 'en', '✍[*❽❽❽*] :: Dragon City hack jaibreak Food | Android/iOS', 1771, 1, 1466776652, 1466776652, 0, 0, 0, 0, 0, 0),
(8030, 8039, 'author', 'en', '☞HACKGENER☦ATOR 24/06/2016 :: Double Dragon Trilogy Juwelen H.a.c.k. generate Health |; 2016', 1771, 1, 1466776656, 1466776656, 0, 0, 0, 0, 0, 0),
(8031, 8040, 'author', 'en', '☥[~�,��|�❼~]☥ :: KENDALL and KYLIE hack For android ', 1771, 1, 1466776661, 1466776661, 0, 0, 0, 0, 0, 0),
(8032, 8041, 'author', 'en', '▬(!❸❸❸!)▬ :: Disney Magic Kingdoms hack tool download generate Gems, Ma❈gic | Android', 1771, 1, 1466776663, 1466776663, 0, 0, 0, 0, 0, 0),
(8033, 8042, 'author', 'en', '♢{!❻❻❻!}♤ :: 3d Bowling hack apk generate Cash, Coins | iOS-And\\roid', 1771, 1, 1466776666, 1466776666, 0, 0, 0, 0, 0, 0),
(8034, 8043, 'author', 'en', '◒[*[❾❾❾]*] :: The Walking Dead Road to Survival ch.eats real hack Coins, MatEriAl | Android/iOS', 1771, 1, 1466776674, 1466776674, 0, 0, 0, 0, 0, 0),
(8035, 8044, 'author', 'en', '✫(*(❻❻❻)*)✬ :: Vainglory tips for noobs Glory, Gold | iOS-Android', 1771, 1, 1466776678, 1466776678, 0, 0, 0, 0, 0, 0),
(8036, 8045, 'author', 'en', '☺|¯\\_�.�.�.�.�.�.�.�.�._/¯|☺ :: Pes Manager hac✠k android no root F;ounds | 2016', 1771, 1, 1466776684, 1466776684, 0, 0, 0, 0, 0, 0),
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(8041, 8050, 'author', 'en', '✯[*[❸❸❸]*]✯ :: Real Steel World Robot Boxing hack ifile Gold | ANdroId/iOS', 1771, 1, 1466776704, 1466776704, 0, 0, 0, 0, 0, 0),
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(8050, 8059, 'author', 'en', '[![❸❸❸*]!] :: Terraria Hack Coins, Health | 2016', 1771, 1, 1466776777, 1466776777, 0, 0, 0, 0, 0, 0),
(8051, 8060, 'author', 'en', '♩{!❾❾❾!}♪ :: Clash Royale tips cheats generaTe Gold,♊ Gems | 2016', 1771, 1, 1466776778, 1466776778, 0, 0, 0, 0, 0, 0),
(8052, 8061, 'author', 'en', '♢GENERATOR 24/06/2016 :: Clash Royale ipa hack Go✭ld | Android/iOS', 1771, 1, 1466776781, 1466776781, 0, 0, 0, 0, 0, 0),
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(8055, 8064, 'author', 'en', '☭{~hack~}☭ 24.06.2016 :: KENDALL and KYLIE Juwelen Hack ', 1771, 1, 1466776799, 1466776799, 0, 0, 0, 0, 0, 0),
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(8057, 8066, 'author', 'en', '▮(*2016*) 24-06-2016 :: NBA LIVE Mobile hack for ios hack Cash | Android', 1771, 1, 1466776811, 1466776811, 0, 0, 0, 0, 0, 0),
(8058, 8067, 'author', 'en', '☒!FULLHACK 24:06:2016 :: NBA LIVE Mobile Cheats generate C▧oins | 2016', 1771, 1, 1466776813, 1466776813, 0, 0, 0, 0, 0, 0),
(8059, 8068, 'author', 'en', '☒{!{❶❶�|�}!} :: KENDALL and KYLIE Astuce ', 1771, 1, 1466776820, 1466776820, 0, 0, 0, 0, 0, 0),
(8060, 8069, 'author', 'en', '✾[*[MAY]*] 24;-06-2016 :: MARVEL Avengers Academy cheat codes generate Credits, Shards | Android', 1771, 1, 1466776822, 1466776822, 0, 0, 0, 0, 0, 0),
(8061, 8070, 'author', 'en', '◶~!~❹❹❹.◷ :: Contract Killer Sniper c♤heat app free Cash | iOS-Android', 1771, 1, 1466776826, 1466776826, 0, 0, 0, 0, 0, 0),
(8062, 8071, 'author', 'en', '✰$hackEngine✰ 24:06:2016 :: Clash Royale Hack 2016 free Gold, Gems | Android', 1771, 1, 1466776834, 1466776834, 0, 0, 0, 0, 0, 0),
(8063, 8072, 'author', 'en', '❃###MEGAHACK :: Monster Warlord hack no survey  Gold, HealTh | Android', 1771, 1, 1466776836, 1466776836, 0, 0, 0, 0, 0, 0),
(8064, 8073, 'author', 'en', '✧❹❹❹✧ :: Adventures Of Mana cheats that actually work Coins | i.O.S.-.A.n.d.r.o.i.d.', 1771, 1, 1466776842, 1466776842, 0, 0, 0, 0, 0, 0),
(8065, 8074, 'author', 'en', '✬{*❹❹❹*}✬ :: iSlash Heroes Generator ', 1771, 1, 1466776843, 1466776843, 0, 0, 0, 0, 0, 0),
(8066, 8075, 'author', 'en', '✳{!{cheats}!}✳ 24:06:2016 :: Taichi Panda hack codes ♫', 1771, 1, 1466776847, 1466776847, 0, 0, 0, 0, 0, 0),
(8067, 8076, 'author', 'en', '☞[*[❹❹.�●�]*]☟ :: Evolution Battle for Utopia hack software Diamond.s | Android/iOS', 1771, 1, 1466776858, 1466776858, 0, 0, 0, 0, 0, 0),
(8068, 8077, 'author', 'en', '●[~❾❾.❾~]◐ :: Ultimate Robot Fighting hack no j◆aIlbreak Goldpoint | Android', 1771, 1, 1466776860, 1466776860, 0, 0, 0, 0, 0, 0),
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(8070, 8079, 'author', 'en', '❝{!{❸❸❸}!}❞ :: The Walking Dead No Man Land hack ifile generate. Xp | 2016', 1771, 1, 1466776865, 1466776865, 0, 0, 0, 0, 0, 0),
(8071, 8080, 'author', 'en', '◛{!❽❽�❆��!}◜ :: Star Trek Timelines cheats ipad generate Credits | 201;6', 1771, 1, 1466776871, 1466776871, 0, 0, 0, 0, 0, 0),
(8072, 8081, 'author', 'en', '❔[ch▪eats]❕ 24:06::2016 :: Grand Theft Auto China Town hack codes gen♀erate Money, Rp | 2016', 1771, 1, 1466776884, 1466776884, 0, 0, 0, 0, 0, 0),
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(8075, 8084, 'author', 'en', '♅{!{cheats}!}♆ 24/06/2016 :: Marvel Super Heroes cheats for iphone ', 1771, 1, 1466776892, 1466776892, 0, 0, 0, 0, 0, 0),
(8076, 8085, 'author', 'en', '✵(!(MAY)!) :: Grand Theft Auto Vice City cheats download f.ree Rp | iOS-Android', 1771, 1, 1466776896, 1466776896, 0, 0, 0, 0, 0, 0),
(8077, 8086, 'author', 'en', '◆[[�◮��❸❸]]◇ :: 3d Bowling hack mo❈d a^p◌k ', 1771, 1, 1466776912, 1466776912, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(8078, 8087, 'author', 'en', '♩fullh..ack♪ :: Dragon Mania Legends hack ios free Gems, Gold | Andro❏id', 1771, 1, 1466776913, 1466776913, 0, 0, 0, 0, 0, 0),
(8079, 8088, 'author', 'en', '☕((❼❼❼)) :: Angry Birds Rio hack free Slingscope | iOS-Android', 1771, 1, 1466776916, 1466776916, 0, 0, 0, 0, 0, 0),
(8080, 8089, 'author', 'en', '◊#HACKGENERATOR◊ :: Clash Royale cheats Free hack Gol☹d, Gems | iOS❊-Android', 1771, 1, 1466776917, 1466776917, 0, 0, 0, 0, 0, 0),
(8081, 8090, 'author', 'en', '○###HACKPACK◌ 24-0♐6✒-2016 :: Clash Royale hacked apk free Gold, Gems | iOS-Android', 1771, 1, 1466776918, 1466776918, 0, 0, 0, 0, 0, 0),
(8082, 8091, 'author', 'en', '❟{HACK} 24:06:201❑6 :: Zombie Catchers cheat engine ', 1771, 1, 1466776934, 1466776934, 0, 0, 0, 0, 0, 0),
(8083, 8092, 'author', 'en', '♀$gethack♁ 2.4./.0.6./.2.0.1.6. :: Lords Mobile cheat real ', 1771, 1, 1466776934, 1466776934, 0, 0, 0, 0, 0, 0),
(8084, 8093, 'author', 'en', '☈{!❾❾❾!}☉ :: Subway Surfers Hack 2016 free Coins | Android/iOS', 1771, 1, 1466776938, 1466776938, 0, 0, 0, 0, 0, 0),
(8085, 8094, 'author', 'en', '♩~!~ENGINE :: Sonic The Hedgehog 2 hack mod free Gold | Android/iOS', 1771, 1, 1466776941, 1466776941, 0, 0, 0, 0, 0, 0),
(8086, 8095, 'author', 'en', '◛¯\\_MAY_/¯ :: Hotel Dash c*heats for mobile ', 1771, 1, 1466776941, 1466776941, 0, 0, 0, 0, 0, 0),
(8087, 8096, 'author', 'en', '✪HACKPACK✪ 24.06.2016 :: Pet Alliance Glitch ', 1771, 1, 1466776954, 1466776954, 0, 0, 0, 0, 0, 0),
(8088, 8097, 'author', 'en', '❐(!❷❷❷!)❑ :: Criminal Case cheats for✺ Iphone free Coins | iOS-Android', 1771, 1, 1466776957, 1466776957, 0, 0, 0, 0, 0, 0),
(8089, 8098, 'author', 'en', '❞_/¯h□ackcheats¯\\_❟ :: World Chef cheats for mobile ', 1771, 1, 1466776958, 1466776958, 0, 0, 0, 0, 0, 0),
(8090, 8099, 'author', 'en', '◟{!{❻❻❻}!} :!:! Domination cheat engine !', 1771, 1, 1466776965, 1466776965, 0, 0, 0, 0, 0, 0),
(8091, 8100, 'author', 'en', '♉[~MEGAHACK~] 24/06/2016 :: Cars Fast As Lightning hack online ', 1771, 1, 1466776966, 1466776966, 0, 0, 0, 0, 0, 0),
(8092, 8101, 'author', 'en', '♧{{❶❶❶}}♧ :: Hotel Transylvania 2 hack no❧ suRvey  hack Coins | iOS-Android', 1771, 1, 1466776988, 1466776988, 0, 0, 0, 0, 0, 0),
(8093, 8102, 'author', 'en', '☫###0..6.....2..4.....2..0..1..6..☬ :: NBA LIVE Mobile hack real f☋reE Coins,, Cash |◸ 2016', 1771, 1, 1466776990, 1466776990, 0, 0, 0, 0, 0, 0),
(8094, 8103, 'author', 'en', '☧$❷❷❷☧ :: Puzzle And Dragons Mod Apk ', 1771, 1, 1466777004, 1466777004, 0, 0, 0, 0, 0, 0),
(8095, 8104, 'author', 'en', '☍(!(❹�.�❹)!)☎ :: Disney Magic Kingdoms Triche◈ ', 1771, 1, 1466777006, 1466777006, 0, 0, 0, 0, 0, 0),
(8096, 8105, 'author', 'en', '✍[[CHEAT]] 24-06-2016 :: Little Empire Hack No Root hack Cristal | Android/iOS', 1771, 1, 1466777007, 1466777007, 0, 0, 0, 0, 0, 0),
(8097, 8106, 'author', 'en', '❁{*❽❽❽*}❂ :: Lords Mobile cheats f.or mob.il♂e free Gems, GOld', 1771, 1, 1466777012, 1466777012, 0, 0, 0, 0, 0, 0),
(8098, 8107, 'author', 'en', '▷|_/¯❹❹❹¯\\_|▸ :: 3d Bowling hack mac hack Cash, Coins | iOS-Android', 1771, 1, 1466777031, 1466777031, 0, 0, 0, 0, 0, 0),
(8099, 8108, 'author', 'en', '✲{!{�\\��❻❻}!} :: c.h.e.a.t. Gangstar Vegas ', 1771, 1, 1466777033, 1466777033, 0, 0, 0, 0, 0, 0),
(8100, 8109, 'author', 'en', '♫HACKENGINE♫ 24-06-2016 :: Clash Royale Cheats 2016 ', 1771, 1, 1466777036, 1466777036, 0, 0, 0, 0, 0, 0),
(8101, 8110, 'author', 'en', '▱[❸|❸❸]▱ :: Clash Royale hack mod apk .✴', 1771, 1, 1466777038, 1466777038, 0, 0, 0, 0, 0, 0),
(8102, 8111, 'author', 'en', '☺{!HACKPACK!} 24.06.2016 :: Clash Royale TrIche Gratuit ', 1771, 1, 1466777039, 1466777039, 0, 0, 0, 0, 0, 0),
(8103, 8112, 'author', 'en', '❁(!(❾❾�^��)!) :: Need For Speed cheat real generate Cas☢h, Fuel |^ iOS-Android', 1771, 1, 1466777053, 1466777053, 0, 0, 0, 0, 0, 0),
(8104, 8113, 'author', 'en', '☾(~❸❸❸~) :: Mobile Strike cheat code entry generate Stone | Android/iO✦S|', 1771, 1, 1466777059, 1466777059, 0, 0, 0, 0, 0, 0),
(8105, 8114, 'author', 'en', '✰[![❹❹❹]!]✰ :: Clash Royale hack no downl.oad  free Gold', 1771, 1, 1466777060, 1466777060, 0, 0, 0, 0, 0, 0),
(8106, 8115, 'author', 'en', '❗(*❷❷❷*)❗ :: BLEACH Brave Souls cheats that work Spirit Orbs | Android', 1771, 1, 1466777064, 1466777064, 0, 0, 0, 0, 0, 0),
(8107, 8116, 'author', 'en', '✂(*(�\\�❹❹)*)✂ :: Clash of Clans cheat real free Gold | Android', 1771, 1, 1466777077, 1466777077, 0, 0, 0, 0, 0, 0),
(8108, 8117, 'author', 'en', '▬$�☱�❷❷ :: Hitman Sniper cheats real Blood|, Money', 1771, 1, 1466777081, 1466777081, 0, 0, 0, 0, 0, 0),
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(8110, 8119, 'author', 'en', '☾{~❼❼❼~} :: MARVEL Avengers Academy Hack Gratis ', 1771, 1, 1466777087, 1466777087, 0, 0, 0, 0, 0, 0),
(8111, 8120, 'author', 'en', '☨{!{❽❽❽}!} :: Clash of Clans cHeats r.eal ', 1771, 1, 1466777087, 1466777087, 0, 0, 0, 0, 0, 0),
(8112, 8121, 'author', 'en', '⚓(*❷❷❷*)⚓ :: Nba 2k15 Pir..ate,r ', 1771, 1, 1466777098, 1466777098, 0, 0, 0, 0, 0, 0),
(8113, 8122, 'author', 'en', '✷[[❹❹❹]]✸ :: Arcane Legend APK IPA Mod ', 1771, 1, 1466777102, 1466777102, 0, 0, 0, 0, 0, 0),
(8114, 8123, 'author', 'en', '♒{!24:06:▣2016!} ::☞ The Walking Dead No Man Land hack online/ f*r*e*e* Xp', 1771, 1, 1466777108, 1466777108, 0, 0, 0, 0, 0, 0),
(8115, 8124, 'author', 'en', '✸(*�✳��❸❸*) :: The Walking Dead Road to Survival Hack Deutsch free Material', 1771, 1, 1466777109, 1466777109, 0, 0, 0, 0, 0, 0),
(8116, 8125, 'author', 'en', '■_/¯gethack¯\\_■ 24:06:2016 :: Ninja Village hack cheats ', 1771, 1, 1466777110, 1466777110, 0, 0, 0, 0, 0, 0),
(8117, 8126, 'author', 'en', '✿{fullhack} :▹: Grand Theft Auto China Town hack telech,arger ', 1771, 1, 1466777119, 1466777119, 0, 0, 0, 0, 0, 0),
(8118, 8127, 'author', 'en', '♵{!❼❼❼!}♶ :: 3d Bowling tips tricks gener.at☡e Coins', 1771, 1, 1466777121, 1466777121, 0, 0, 0, 0, 0, 0),
(8119, 8128, 'author', 'en', '☓{{h\\ack}}☔ 24-06-2016 :: Clash Royale cheatS and ha☎cks gene♞ratE Gold, Gems. | 2016', 1771, 1, 1466777133, 1466777133, 0, 0, 0, 0, 0, 0),
(8120, 8129, 'author', 'en', '♢~!~h❙ackpack :: Clash Royale cheats download hack Gold | iOS-A✬ndroid', 1771, 1, 1466777134, 1466777134, 0, 0, 0, 0, 0, 0),
(8121, 8130, 'author', 'en', '▻❼❼❼▼ :: Shadow Gun hack for anDroid Cash, Gold', 1771, 1, 1466777135, 1466777135, 0, 0, 0, 0, 0, 0),
(8122, 8131, 'author', 'en', '❚MOD❚ 24.06.2016 :: Adventures Of Mana tips and tricks generate Coins, Hp |♉ Android/iOS', 1771, 1, 1466777141, 1466777141, 0, 0, 0, 0, 0, 0),
(8123, 8132, 'author', 'en', '◒¯\\_online_/¯◓ 24.06.20.16♉ :: Grand Theft Auto III GliTch and Gli■tches Money | Android', 1771, 1, 1466777146, 1466777146, 0, 0, 0, 0, 0, 0),
(8124, 8133, 'author', 'en', '❑❾❾❾.❒ :: MARVEL Avengers Academy hack tool download Credits, Shards | Android/i.OS', 1771, 1, 1466777155, 1466777155, 0, 0, 0, 0, 0, 0),
(8125, 8134, 'author', 'en', '☛❶❶❶ :: Csr Racing Online Hack free GoLd | 2016', 1771, 1, 1466777158, 1466777158, 0, 0, 0, 0, 0, 0),
(8126, 8135, 'author', 'en', '❕|¯\\_hack_/¯| 24-06-2016 :: Terraria hack no survey  genera/te Coins | 2016', 1771, 1, 1466777160, 1466777160, 0, 0, 0, 0, 0, 0),
(8127, 8136, 'author', 'en', '(!(❸�.�❸)!) :: Kingsroad cheats that Work ', 1771, 1, 1466777164, 1466777164, 0, 0, 0, 0, 0, 0),
(8128, 8137, 'author', 'en', '♔(❸❸❸)♔ :: Mobile Strike hack software ', 1771, 1, 1466777166, 1466777166, 0, 0, 0, 0, 0, 0),
(8129, 8138, 'author', 'en', '☥{!{❺❺❺}!}☥ :: Ramboat hack no root free Gems', 1771, 1, 1466777176, 1466777176, 0, 0, 0, 0, 0, 0),
(8130, 8139, 'author', 'en', '▰❺❺❺▰ :: Fatal Fury Special Cheats 2016 hack Coins, Health | Android/iOS', 1771, 1, 1466777180, 1466777180, 0, 0, 0, 0, 0, 0),
(8131, 8140, 'author', 'en', '☘❶❶❶☘ :: Terraria tips❎ for. beginners Coins, Health', 1771, 1, 1466777182, 1466777182, 0, 0, 0, 0, 0, 0),
(8132, 8141, 'author', 'en', '♈(❻❻.❻)♉ :: no survey hack google play Contract Killer 2  free Cash | Android', 1771, 1, 1466777185, 1466777185, 0, 0, 0, 0, 0, 0),
(8133, 8142, 'author', 'en', '♪[~[❾❾�,��]~]♪ :: Sniper Swat Fps Hack Tool APK IPA hack Gold | AndroId', 1771, 1, 1466777186, 1466777186, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(8403, 8412, 'author', 'en', '▪{!❾❾❾!}▪ :: Jelly Defense hack real Coins | Android', 1771, 1, 1466778519, 1466778519, 0, 0, 0, 0, 0, 0),
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(8679, 8688, 'author', 'en', '◊(!HACKT☈OOL!) 24:06:2016 :: Modern Combat 5 Blackout Hack No Su|rvey Gol♼dcredits | iO♷S-Android♵', 1771, 1, 1466779899, 1466779899, 0, 0, 0, 0, 0, 0),
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(8681, 8690, 'author', 'en', '◭[~❹❹❹~] :: Marvel Pinball hack ch.eats ..', 1771, 1, 1466779913, 1466779913, 0, 0, 0, 0, 0, 0),
(8682, 8691, 'author', 'en', '►¯\\_2016_/¯▻ 24:06:2016 :: Clash Royale hack mac hack Gold | iOS-Android', 1771, 1, 1466779917, 1466779917, 0, 0, 0, 0, 0, 0),
(8683, 8692, 'author', 'en', '✩(~HACKENGINE~)✩ 24/06/2016◧ :: KENDALL and KYLIE hack no jaibreak hack Gems | iOS-Android', 1771, 1, 1466779920, 1466779920, 0, 0, 0, 0, 0, 0),
(8684, 8693, 'author', 'en', '☛{~{❺�◺�❺}~} :: Clash Royale hack software free Gold, Gems', 1771, 1, 1466779921, 1466779921, 0, 0, 0, 0, 0, 0),
(8685, 8694, 'author', 'en', '◤#06/24/2016◤ :: The Walking Dead Road to Survival hack software Coins, Material | 2016', 1771, 1, 1466779927, 1466779927, 0, 0, 0, 0, 0, 0),
(8686, 8695, 'author', 'en', '◣[~[hackcheats]~]◣ :: Clash of Clans cheats and tips❞ /', 1771, 1, 1466779933, 1466779933, 0, 0, 0, 0, 0, 0),
(8687, 8696, 'author', 'en', '☥(!(GENERATOR)!)☦ 24.06.2016 :: Clash Royale hack tool free Gems | iOS-Android', 1771, 1, 1466779936, 1466779936, 0, 0, 0, 0, 0, 0),
(8688, 8697, 'author', 'en', '▣$2016▤ 24.06.2016 :: C✹o◸de de TRiChe Dragon City generat♄e Food, Gold', 1771, 1, 1466779942, 1466779942, 0, 0, 0, 0, 0, 0),
(8689, 8698, 'author', 'en', '♞(!(❺❺❺)!) :: Garoumark Of The Wolves hack for android Gold | 2016', 1771, 1, 1466779947, 1466779947, 0, 0, 0, 0, 0, 0),
(8690, 8699, 'author', 'en', '✨¯\\_2016_/¯ 24-06-2016 :: Clash Royale cheat engine ', 1771, 1, 1466779953, 1466779953, 0, 0, 0, 0, 0, 0),
(8691, 8700, 'author', 'en', '❃[~[GETHACK]~]❅ 24:06◠:2016 :: Adventures Of Mana hack for ios generate Coins | iOS-Android◧', 1771, 1, 1466779957, 1466779957, 0, 0, 0, 0, 0, 0),
(8692, 8701, 'author', 'en', '☨{ONLINE}☩ 24:06:2016 :: KENDALL and KYLIE hack tool ', 1771, 1, 1466779963, 1466779963, 0, 0, 0, 0, 0, 0),
(8693, 8702, 'author', 'en', '☷!engine☸ 24/06/2016 :: The Walking Dead Pinball hack that works ', 1771, 1, 1466779968, 1466779968, 0, 0, 0, 0, 0, 0),
(8694, 8703, 'author', 'en', '✯~!~❷❷❷✪ :: Ramboat hack actiVation code generate Coins | Android', 1771, 1, 1466779968, 1466779968, 0, 0, 0, 0, 0, 0),
(8695, 8704, 'author', 'en', '✵(*Hackengine*) 24-06-2016 :: Clash Royale hack❇ tool generator generate Gems | Android', 1771, 1, 1466779974, 1466779974, 0, 0, 0, 0, 0, 0),
(8696, 8705, 'author', 'en', '☔$�✰�❼❼.☔ :: Goat Simulator cheat that works hack Gold, Power', 1771, 1, 1466779978, 1466779978, 0, 0, 0, 0, 0, 0),
(8697, 8706, 'author', 'en', '☧(*❺❺◟❺*)☧ :: Temple Run 2 che◍a,ts for ipad Coins,▿ Gems | iOS-Android', 1771, 1, 1466779993, 1466779993, 0, 0, 0, 0, 0, 0),
(8698, 8707, 'author', 'en', '▶~!~❼❼❼▷ :: Clash Royale Online G!enerator Gold | iOS-Android', 1771, 1, 1466779994, 1466779994, 0, 0, 0, 0, 0, 0),
(8699, 8708, 'author', 'en', '♦{{cheats}} 24:06:2016 :: KENDALL and KYLIE Hack iPhone iPad Android ', 1771, 1, 1466780002, 1466780002, 0, 0, 0, 0, 0, 0),
(8700, 8709, 'author', 'en', '❓(~(❷❷❷)~)❔ :: World Chef Pir♿ater Gems | iOS-Android', 1771, 1, 1466780007, 1466780007, 0, 0, 0, 0, 0, 0),
(8701, 8710, 'author', 'en', '◆|¯\\_❹❹❹_/¯|◇ ::! Batlle Camp Trucos Energ.☖y | Android/iOS', 1771, 1, 1466780018, 1466780018, 0, 0, 0, 0, 0, 0),
(8702, 8711, 'author', 'en', '♓{!{mod}!} :: Happy Wheels hack telecharger Cur,rency♕, Gold | 2016', 1771, 1, 1466780020, 1466780020, 0, 0, 0, 0, 0, 0),
(8703, 8712, 'author', 'en', '©❼❼❼ :: Mobile Strike Astuce hack Gold, Stone | 2016', 1771, 1, 1466780021, 1466780021, 0, 0, 0, 0, 0, 0),
(8704, 8713, 'author', 'en', '☢[~HACK~]☢ 24-06-2016 :: Monster Legend ch\\eats free hack Gold | Andr:o.id/iOS', 1771, 1, 1466780026, 1466780026, 0, 0, 0, 0, 0, 0),
(8705, 8714, 'author', 'en', '♄(*(❾❾❾)*)♄ :: Clash Royale cheats hacking generate Gold, Gems | iOS-Android', 1771, 1, 1466780032, 1466780032, 0, 0, 0, 0, 0, 0),
(8706, 8715, 'author', 'en', '▻{*❹\\❹❹❚*}▻ :: Respawnables hack codes Cash, Gold', 1771, 1, 1466780046, 1466780046, 0, 0, 0, 0, 0, 0),
(8707, 8716, 'author', 'en', '❃{!❽❽❽!} :: Boom Beach hack tool ', 1771, 1, 1466780048, 1466780048, 0, 0, 0, 0, 0, 0),
(8708, 8717, 'author', 'en', '❂(*MEGAHACK*)❂ 24/06/2016 :: Little Empire hack ifile Gems | iOS-Android', 1771, 1, 1466780049, 1466780049, 0, 0, 0, 0, 0, 0),
(8709, 8718, 'author', 'en', '﨏(!ge.thack!)﨏 :: Punch Boxing 3d cheats hacks ', 1771, 1, 1466780056, 1466780056, 0, 0, 0, 0, 0, 0),
(8710, 8719, 'author', 'en', '♳{~{❽❽❽}~}♳ ::◆ Gangstar Vegas Triche free Key | 2016', 1771, 1, 1466780073, 1466780073, 0, 0, 0, 0, 0, 0),
(8711, 8720, 'author', 'en', '▴{{�^�^�^�^�^�^�^�^�^}} :: Fruit Ninja cheats hacking Fruit, J!u!i!c!e!', 1771, 1, 1466780073, 1466780073, 0, 0, 0, 0, 0, 0),
(8712, 8721, 'author', 'en', '◌~!~❸❸❸◍ :: Shards Of Magic tips and cheats generat|e F.o.o.d.s.', 1771, 1, 1466780076, 1466780076, 0, 0, 0, 0, 0, 0),
(8713, 8722, 'author', 'en', '⚓¯\\_hackpack_/¯⚓ :: Beach Buggy Racing ha.ck free hack Gems | Android◄/iOS', 1771, 1, 1466780076, 1466780076, 0, 0, 0, 0, 0, 0),
(8714, 8723, 'author', 'en', '☳(~(ONL:INE)~)☳ 24.06.2016 :: Hotel Dash tips and cheats hack Cash | iOS-Android', 1771, 1, 1466780080, 1466780080, 0, 0, 0, 0, 0, 0),
(8715, 8724, 'author', 'en', '◟[[❹❹❹]]◟ :: BLEACH Brave Souls che/ats f|o|r| mobile ', 1771, 1, 1466780096, 1466780096, 0, 0, 0, 0, 0, 0),
(8716, 8725, 'author', 'en', '☡{{hackpack}}☢ 24/06/2016 :: Clash Royale hack ios ', 1771, 1, 1466780096, 1466780096, 0, 0, 0, 0, 0, 0),
(8717, 8726, 'author', 'en', '♼(!❸^❸❸!)♽ :: Boom Beach Hack Cheat JuWelen Deutsch! ', 1771, 1, 1466780102, 1466780102, 0, 0, 0, 0, 0, 0),
(8718, 8727, 'author', 'en', '◣[!❷❷❷!]◤ :: Mobile Strike cheat a✓pk generate Stone | iOS-Andr♜o!id', 1771, 1, 1466780102, 1466780102, 0, 0, 0, 0, 0, 0),
(8719, 8728, 'author', 'en', '◞[*[2016]*]◟ :: NBA LIVE Mobile hack mod free Coins, C♛ash', 1771, 1, 1466780104, 1466780104, 0, 0, 0, 0, 0, 0),
(8720, 8729, 'author', 'en', '✩{*{❷❷❷}*}✓ :: Hungry Shark Evolution Hack No R*o*o*t* ', 1771, 1, 1466780118, 1466780118, 0, 0, 0, 0, 0, 0),
(8721, 8730, 'author', 'en', '✺CHEAT✺ 24..-06-2016 :: Clash Royale hack free Gold | A,n,d,r,o,i,d,/,i,O,S,', 1771, 1, 1466780118, 1466780118, 0, 0, 0, 0, 0, 0),
(8722, 8731, 'author', 'en', '✠[[❹❹❹]]✡ :: Badminton 3d hack apk C✯oins |. iOS-Android', 1771, 1, 1466780127, 1466780127, 0, 0, 0, 0, 0, 0),
(8723, 8732, 'author', 'en', '◇(GETHACK;)◇ 24/06/2016 :: Paradise Bay c.h.e.a.t.s. for iphone ', 1771, 1, 1466780130, 1466780130, 0, 0, 0, 0, 0, 0),
(8724, 8733, 'author', 'en', '◞|¯\\_hackpack_/¯|◞ 24.06.2016 :*:* Clash Royale tips and tricks ', 1771, 1, 1466780141, 1466780141, 0, 0, 0, 0, 0, 0),
(8725, 8734, 'author', 'en', '♯[~[❸❸❸]~] :: Hay Day cheats android ', 1771, 1, 1466780143, 1466780143, 0, 0, 0, 0, 0, 0),
(8726, 8735, 'author', 'en', '☓[![�✣�❺❺]!] :: Archery Master 3d H.a.c.k. No Roo|t ', 1771, 1, 1466780148, 1466780148, 0, 0, 0, 0, 0, 0),
(8727, 8736, 'author', 'en', '™[[engine]]™ :: Double Dragon Trilogy HacK ifile generate Health |☧ 2016', 1771, 1, 1466780153, 1466780153, 0, 0, 0, 0, 0, 0),
(8728, 8737, 'author', 'en', '☔[![�▻�❶❶]!]☔ :: KENDALL and KYLIE hack no jaibr.e❘ak ', 1771, 1, 1466780156, 1466780156, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(8729, 8738, 'author', 'en', '✍{!❾❾❾!} :: Evolution Battle for Utopia cheat codes generate Gem,s', 1771, 1, 1466780164, 1466780164, 0, 0, 0, 0, 0, 0),
(8730, 8739, 'author', 'en', '❱❽�\\��❽ :: Clash of Clans hack dOwnlo◂ad free Gems, Gold', 1771, 1, 1466780167, 1466780167, 0, 0, 0, 0, 0, 0),
(8731, 8740, 'author', 'en', '♑(~(C.HEAT)~)♑ 24/06/2016 :: Clash Royale Hack G.ratis generate Gold, GemS', 1771, 1, 1466780168, 1466780168, 0, 0, 0, 0, 0, 0),
(8732, 8741, 'author', 'en', '◻_/¯❸❸❸¯\\_◻ :: MARVEL Avengers Academy hack cheats free Shards |. iOS-Android', 1771, 1, 1466780174, 1466780174, 0, 0, 0, 0, 0, 0),
(8733, 8742, 'author', 'en', '▽|¯\\_❶❶❶_/¯|▾ :: Clash Royale hack for ios generate Gold | Android/iOS', 1771, 1, 1466780181, 1466780181, 0, 0, 0, 0, 0, 0),
(8734, 8743, 'author', 'en', '✸❹❹❹✹ :: Hotel Transylvania 2 Hacker ', 1771, 1, 1466780186, 1466780186, 0, 0, 0, 0, 0, 0),
(8735, 8744, 'author', 'en', '☶[*❶❶❶*] :: GTA 5 Pirater free Level | Andro.id/iOS', 1771, 1, 1466780189, 1466780189, 0, 0, 0, 0, 0, 0),
(8736, 8745, 'author', 'en', '﨏|_/¯❹❹❹¯\\_| :: Clash Royale tips and cheats f☈ree Gold, Gems', 1771, 1, 1466780190, 1466780190, 0, 0, 0, 0, 0, 0),
(8737, 8746, 'author', 'en', '☫[!❷❷❷!]☫ :: BLEACH Brave Souls apk mega mod generate Spirit Orbs, Gold | iOS-AndroiD', 1771, 1, 1466780197, 1466780197, 0, 0, 0, 0, 0, 0),
(8738, 8747, 'author', 'en', '❊{!{cheat}!}❊ 24-06-2016 :: Clash Royale hack that works ', 1771, 1, 1466780201, 1466780201, 0, 0, 0, 0, 0, 0),
(8739, 8748, 'author', 'en', '♮[![❸❸❸]!]♮ :☘: Bubble Witch 2 Saga hack app◵ Coins, Gold | 2016', 1771, 1, 1466780206, 1466780206, 0, 0, 0, 0, 0, 0),
(8740, 8749, 'author', 'en', '✴(!(GENERATOR)!)✵ 24.06.2016 :: Battlefront Heroes hack safe✳ ', 1771, 1, 1466780211, 1466780211, 0, 0, 0, 0, 0, 0),
(8741, 8750, 'author', 'en', '○HACKTOOL○ 24:06:2016 :: Dragon Hills Triche GratuIt ', 1771, 1, 1466780219, 1466780219, 0, 0, 0, 0, 0, 0),
(8742, 8751, 'author', 'en', '♫[[CHEATS]]♬ 24-06-20✤16 ::| Clash Royale cheats for iphone Gold, Gems | 2✉016', 1771, 1, 1466780226, 1466780226, 0, 0, 0, 0, 0, 0),
(8743, 8752, 'author', 'en', '◆(!❽❽❽!) :: Clash Royale h♆ack jaibreak ', 1771, 1, 1466780228, 1466780228, 0, 0, 0, 0, 0, 0),
(8744, 8753, 'author', 'en', '☤$❼❼❼ :: how to geT hack tool Clash Royale generate Gold■ | 2..016', 1771, 1, 1466780233, 1466780233, 0, 0, 0, 0, 0, 0),
(8745, 8754, 'author', 'en', '☼|¯\\_hackpack_/¯| 24:06:2016 :: Dragon City hack activation code generate Food,. G.o.l.d. | Android', 1771, 1, 1466780241, 1466780241, 0, 0, 0, 0, 0, 0),
(8746, 8755, 'author', 'en', '✻[~❹❹❹◟~] :: Samurai Siege Mod Apk free Diamon☡d | 2016', 1771, 1, 1466780250, 1466780250, 0, 0, 0, 0, 0, 0),
(8747, 8756, 'author', 'en', '❍[[�/�/�/�/�/�/�/�/�/]] :: NBA LIVE Mobile hack apk f.ree Coins, Cash | iOS-Android', 1771, 1, 1466780253, 1466780253, 0, 0, 0, 0, 0, 0),
(8748, 8757, 'author', 'en', '▪MAY▫ 24-06-2016 :: Fast Furious Legacy Hack Cheat Juwelen De❒utsch! fre/e Fuel', 1771, 1, 1466780255, 1466780255, 0, 0, 0, 0, 0, 0),
(8749, 8758, 'author', 'en', '❋{!{megahack}!} 24/06/2016 :: Shadow Fight 2 Hack Gratis free Rubies', 1771, 1, 1466780257, 1466780258, 0, 0, 0, 0, 0, 0),
(8750, 8759, 'author', 'en', '◞[!❷❷❷!]◞ :: Dino Hunter As.tuce ', 1771, 1, 1466780270, 1466780270, 0, 0, 0, 0, 0, 0),
(8751, 8760, 'author', 'en', '✾(!❶❶❶!) ::♺ how to hacK Dawn Of The Sniper hack Cash, Coins | 2016', 1771, 1, 1466780275, 1466780275, 0, 0, 0, 0, 0, 0),
(8752, 8761, 'author', 'en', '❐(!❷❷❷!)❐ :: Farmville 2 hack tool generate Coins | Android/iOS', 1771, 1, 1466780277, 1466780277, 0, 0, 0, 0, 0, 0),
(8753, 8762, 'author', 'en', '✶###❶❶❶ :♉: NBA LIVE Mobile cheat codes .', 1771, 1, 1466780280, 1466780280, 0, 0, 0, 0, 0, 0),
(8754, 8763, 'author', 'en', '♺[~�..�..�..�..�..�..�..�..�..~]♻ :: Fishing Paradise 3d ch®eats ip|h.one hack Coins | Android/iOS', 1771, 1, 1466780288, 1466780288, 0, 0, 0, 0, 0, 0),
(8755, 8764, 'author', 'en', '◝¯\\_HACK*ED▦_/¯◞ 2▩4/06/2016◨ :: how to hack Badminton 3d generaT▩e Ball, Coins | 2016', 1771, 1, 1466780293, 1466780293, 0, 0, 0, 0, 0, 0),
(8756, 8765, 'author', 'en', '◧(~(❻❻❻)~) :: Real Football 2013 pirater telecharger ', 1771, 1, 1466780298, 1466780298, 0, 0, 0, 0, 0, 0),
(8757, 8766, 'author', 'en', '◐(!cheat!)◑ 24:06:2016 :: The Walking Dead Road to Survival pirater tele..charger hack Material | Android/iOS', 1771, 1, 1466780300, 1466780300, 0, 0, 0, 0, 0, 0),
(8758, 8767, 'author', 'en', '☽❶❶❶☽ :: Shadow Fight 2 cheat apk hack Coins | iOS-And❊roid', 1771, 1, 1466780310, 1466780310, 0, 0, 0, 0, 0, 0),
(8759, 8768, 'author', 'en', '♬#engine :: Killer Zombie cheats for ipad generate Ge;ms | iOS-Android', 1771, 1, 1466780316, 1466780316, 0, 0, 0, 0, 0, 0),
(8760, 8769, 'author', 'en', '✼GETHACK✽ 2|4-06-2016 :: MARVEL Avengers Academy cheats hacks Credits | iOS▼-Android', 1771, 1, 1466780322, 1466780322, 0, 0, 0, 0, 0, 0),
(8761, 8770, 'author', 'en', '◸[*[❷❷❷]*] :: Deer Hunter 2016 cheats hacking generate Gold | Android', 1771, 1, 1466780324, 1466780324, 0, 0, 0, 0, 0, 0),
(8762, 8771, 'author', 'en', '☙{*{❷❷❷}*}☚ :: Fruit Ninja Hack Deutsch free Fruit, Juice | Android', 1771, 1, 1466780324, 1466780324, 0, 0, 0, 0, 0, 0),
(8763, 8772, 'author', 'en', '▭[~[HACK]~]▮ 24/06/2016 :: Clash Royale Hack Gratis g♞enerate GolD | 2016', 1771, 1, 1466780337, 1466780337, 0, 0, 0, 0, 0, 0),
(8764, 8773, 'author', 'en', '◩{*{fullhack}*}◩ :: Gangstar Vegas hack no root ', 1771, 1, 1466780337, 1466780337, 0, 0, 0, 0, 0, 0),
(8765, 8774, 'author', 'en', '❞[!❾�.��❾!] :: Telecharger Clash of Clans Hack Gratuit hack Gems | 2016', 1771, 1, 1466780344, 1466780344, 0, 0, 0, 0, 0, 0),
(8766, 8775, 'author', 'en', '❞#❶❶❶ :: Hayday c◂heat code entry free Diamond | iOS-Android', 1771, 1, 1466780346, 1466780346, 0, 0, 0, 0, 0, 0),
(8767, 8776, 'author', 'en', '▰$❹❹❹▱ :: BLEACH Brave Souls cheats real hack Go.ld | Android', 1771, 1, 1466780348, 1466780348, 0, 0, 0, 0, 0, 0),
(8768, 8777, 'author', 'en', '◷(!(ONLINE)!)◸ 24/06/2016 :: Top Eleven 2015 Hack Cheats Trainer ', 1771, 1, 1466780361, 1466780361, 0, 0, 0, 0, 0, 0),
(8769, 8778, 'author', 'en', '♈❾❾❾; :: Basketball Stars HAc♝k 2016 Gold, Cash | 2016', 1771, 1, 1466780363, 1466780363, 0, 0, 0, 0, 0, 0),
(8770, 8779, 'author', 'en', '▰|¯\\_gethack_/¯|▱ 24:06:2016◓ :: Vector tips advanced♌ ', 1771, 1, 1466780369, 1466780369, 0, 0, 0, 0, 0, 0),
(8771, 8780, 'author', 'en', '☾[![❼❼❼]!] :: Six Guns H◥ack Cheats Trainer ', 1771, 1, 1466780371, 1466780371, 0, 0, 0, 0, 0, 0),
(8772, 8781, 'author', 'en', '❉[[HACKGENERATOR]]❉ :: Sonic The Hedgehog 2 Hack☿ for iphone ', 1771, 1, 1466780372, 1466780372, 0, 0, 0, 0, 0, 0),
(8773, 8782, 'author', 'en', '[!❻❻❻!] :: Clash Royale tips cheats ', 1771, 1, 1466780387, 1466780387, 0, 0, 0, 0, 0, 0),
(8774, 8783, 'author', 'en', '▬24./06/2016 :: Evolution Battle for Utopia hack android no root Diamonds, Gems', 1771, 1, 1466780392, 1466780392, 0, 0, 0, 0, 0, 0),
(8775, 8784, 'author', 'en', '♒(!FULLHACK!)♒ 24:06:2016 :: Clash Royale cheat apk free Gold', 1771, 1, 1466780395, 1466780395, 0, 0, 0, 0, 0, 0),
(8776, 8785, 'author', 'en', '♕((hackcheats))♕ 24.06.2016 :: Clash Royale apk me✻ga mod Hack Gems |, 2016', 1771, 1, 1466780397, 1466780397, 0, 0, 0, 0, 0, 0),
(8777, 8786, 'author', 'en', '✾[*[❺❺❺]*] :❇: Clash of Clans cheats tool Generate Gems, Gold | Android/iOS', 1771, 1, 1466780410, 1466780410, 0, 0, 0, 0, 0, 0),
(8778, 8787, 'author', 'en', '☭(*❽❽❽*)☭ :: World Conqueror 3 Triche Gra☶tuit ', 1771, 1, 1466780411, 1466780411, 0, 0, 0, 0, 0, 0),
(8779, 8788, 'author', 'en', '◆online :: Habbo Triche free CrEdits | Android/iOS', 1771, 1, 1466780417, 1466780417, 0, 0, 0, 0, 0, 0),
(8780, 8789, 'author', 'en', '✓(~❼❼❼~) :: Batlle Camp cheat review Energy | iOS-Android', 1771, 1, 1466780419, 1466780419, 0, 0, 0, 0, 0, 0),
(8781, 8790, 'author', 'en', '❏[![❻❻❻]!] :: Star Wars Galaxy of Heroes cheats for ipad. ', 1771, 1, 1466780422, 1466780422, 0, 0, 0, 0, 0, 0),
(8782, 8791, 'author', 'en', '◛[~MAY:~] :: Hay Day cheat download hack Diamonds', 1771, 1, 1466780434, 1466780434, 0, 0, 0, 0, 0, 0),
(8783, 8792, 'author', 'en', '♥[~[hackpack]~] 24/06/2016 :: how| to hack Zombie Catchers ', 1771, 1, 1466780434, 1466780434, 0, 0, 0, 0, 0, 0),
(8784, 8793, 'author', 'en', '♗{*{❾❾�.��}*} :: Clash Royale hack real Gems | 2016', 1771, 1, 1466780438, 1466780438, 0, 0, 0, 0, 0, 0),
(8785, 8794, 'author', 'en', '▼[![❷�▻�❷]!] :,: Subway Surfers cheats apk free, Coins, K!eys | 2016', 1771, 1, 1466780439, 1466780439, 0, 0, 0, 0, 0, 0),
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(13357, 13366, 'author', 'en', 'No Script Emsam Pill Glendale', 1888, 1, 1467523482, 1467523482, 0, 0, 0, 0, 0, 0),
(13358, 13367, 'author', 'en', 'sadasda', 1820, 1, 1467525371, 1467525371, 0, 0, 0, 0, 0, 0),
(13359, 13368, 'author', 'en', 'Soccer~:Yanbian Fude vs Hangzhou L ive S treaming O nline', 1820, 1, 1467525553, 1467525553, 0, 0, 0, 0, 0, 0),
(13360, 13369, 'author', 'en', 'Soccer~:Stallions vs PSG L ive S treaming O nline', 1820, 1, 1467525554, 1467525554, 0, 0, 0, 0, 0, 0),
(13361, 13370, 'author', 'en', 'Soccer~:Meizhou Hakka vs Guizhou Zhicheng L ive S treaming O nline', 1820, 1, 1467525555, 1467525555, 0, 0, 0, 0, 0, 0),
(13362, 13371, 'author', 'en', 'Soccer~:Fagiano Okayama vs Shimizu S-Pulse L ive S treaming O nline', 1820, 1, 1467525557, 1467525557, 0, 0, 0, 0, 0, 0),
(13363, 13372, 'author', 'en', 'Soccer~:Giravanz Kitakyushu vs Tokyo Verdy L ive S treaming O nline', 1820, 1, 1467525557, 1467525557, 0, 0, 0, 0, 0, 0),
(13364, 13373, 'author', 'en', 'Soccer~:Mito Hollyhock vs Matsumoto Yamaga L ive S treaming O nline', 1820, 1, 1467525557, 1467525557, 0, 0, 0, 0, 0, 0),
(13365, 13374, 'author', 'en', 'Soccer~:Incheon United vs Jeju United L ive S treaming O nline', 1820, 1, 1467525559, 1467525559, 0, 0, 0, 0, 0, 0),
(13366, 13375, 'author', 'en', 'Soccer~:Montedio Yamagata vs Renofa Yamaguchi L ive S treaming O nline', 1820, 1, 1467525560, 1467525560, 0, 0, 0, 0, 0, 0),
(13367, 13376, 'author', 'en', 'Soccer~:Roasso Kumamoto vs Cerezo Osaka L ive S treaming O nline', 1820, 1, 1467525561, 1467525561, 0, 0, 0, 0, 0, 0),
(13368, 13377, 'author', 'en', 'Soccer~:Tokushima Vortis vs V-Varen Nagasaki L ive S treaming O nline', 1820, 1, 1467525562, 1467525562, 0, 0, 0, 0, 0, 0),
(13369, 13378, 'author', 'en', 'Soccer~:Zweigen Kanazawa vs Kamatamare Sanuki L ive S treaming O nline', 1820, 1, 1467525562, 1467525562, 0, 0, 0, 0, 0, 0),
(13370, 13379, 'author', 'en', 'Soccer~:Quang Nam vs Ha Noi L ive S treaming O nline', 1820, 1, 1467527195, 1467527195, 0, 0, 0, 0, 0, 0),
(13371, 13380, 'author', 'en', 'Soccer~:Da Nang vs Flc Thanh Hoa L ive S treaming O nline', 1820, 1, 1467527195, 1467527195, 0, 0, 0, 0, 0, 0),
(13372, 13381, 'author', 'en', 'Soccer~:Ehime vs JEF United L ive S treaming O nline', 1820, 1, 1467527196, 1467527196, 0, 0, 0, 0, 0, 0),
(13373, 13382, 'author', 'en', 'Soccer~:Gifu vs Kyoto Sanga L ive S treaming O nline', 1820, 1, 1467527197, 1467527197, 0, 0, 0, 0, 0, 0),
(13374, 13383, 'author', 'en', 'Soccer~:Gwangju vs Pohang Steelers L ive S treaming O nline', 1820, 1, 1467527197, 1467527197, 0, 0, 0, 0, 0, 0),
(13375, 13384, 'author', 'en', 'Soccer~:Suwon City vs Jeonbuk Motors L ive S treaming O nline', 1820, 1, 1467527198, 1467527198, 0, 0, 0, 0, 0, 0),
(13376, 13385, 'author', 'en', 'Soccer~:Can Tho vs Hoang Anh Gia Lai L ive S treaming O nline', 1820, 1, 1467527200, 1467527200, 0, 0, 0, 0, 0, 0),
(13377, 13386, 'author', 'en', 'Soccer~:Sanna Khanh Hoa vs Binh Duong L ive S treaming O nline', 1820, 1, 1467527200, 1467527200, 0, 0, 0, 0, 0, 0),
(13378, 13387, 'author', 'en', 'Soccer~:Hai Phong vs Dong Thap L ive S treaming O nline', 1820, 1, 1467527201, 1467527201, 0, 0, 0, 0, 0, 0),
(13379, 13388, 'author', 'en', 'Soccer~:Bahrain vs Jordan L ive S treaming O nline', 1820, 1, 1467527202, 1467527202, 0, 0, 0, 0, 0, 0),
(13380, 13389, 'author', 'en', 'Soccer~:Kazakhstan Beach vs Serbia Beach L ive S treaming O nline', 1820, 1, 1467527203, 1467527203, 0, 0, 0, 0, 0, 0),
(13381, 13390, 'author', 'en', 'Soccer~:Kazakhstan vs Serbia L ive S treaming O nline', 1820, 1, 1467527203, 1467527203, 0, 0, 0, 0, 0, 0),
(13382, 13391, 'author', 'en', 'Soccer~:ThespaKusatsu Gunma vs Machida Zelvia L ive S treaming O nline', 1820, 1, 1467527206, 1467527206, 0, 0, 0, 0, 0, 0),
(13383, 13392, 'author', 'en', 'Soccer~:Song Lam Nghe An vs Quang Ninh L ive S treaming O nline', 1820, 1, 1467527208, 1467527208, 0, 0, 0, 0, 0, 0),
(13384, 13393, 'author', 'en', 'Soccer~:Tusker vs Posta Rangers L ive S treaming O nline', 1820, 1, 1467527209, 1467527209, 0, 0, 0, 0, 0, 0),
(13385, 13394, 'author', 'en', 'Soccer~:Chainat Hornbill vs Sisaket L ive S treaming O nline', 1820, 1, 1467527212, 1467527212, 0, 0, 0, 0, 0, 0),
(13386, 13395, 'author', 'en', 'Soccer~:Air Force Central vs Rayong L ive S treaming O nline', 1820, 1, 1467527215, 1467527215, 0, 0, 0, 0, 0, 0),
(13387, 13396, 'author', 'en', 'Soccer~:Sukhothai vs Chonburi L ive S treaming O nline', 1820, 1, 1467527215, 1467527215, 0, 0, 0, 0, 0, 0),
(13388, 13397, 'author', 'en', 'Soccer~:Lampang vs Krabi L ive S treaming O nline', 1820, 1, 1467527216, 1467527216, 0, 0, 0, 0, 0, 0),
(13389, 13398, 'author', 'en', 'Soccer~:Thai Honda vs Samut Songkhram L ive S treaming O nline', 1820, 1, 1467527219, 1467527219, 0, 0, 0, 0, 0, 0),
(13390, 13399, 'author', 'en', 'Soccer~:Ha Noi T&T vs Long An L ive S treaming O nline', 1820, 1, 1467527220, 1467527220, 0, 0, 0, 0, 0, 0),
(13391, 13400, 'author', 'en', 'Soccer~:KRA vs SoNy Sugar L ive S treaming O nline', 1820, 1, 1467527222, 1467527222, 0, 0, 0, 0, 0, 0),
(13392, 13401, 'author', 'en', 'Soccer~:Estonia Beach vs Moldova Beach L ive S treaming O nline', 1820, 1, 1467527222, 1467527222, 0, 0, 0, 0, 0, 0),
(13393, 13402, 'author', 'en', 'Soccer~:Estonia vs Moldova L ive S treaming O nline', 1820, 1, 1467527224, 1467527224, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(13394, 13403, 'author', 'en', 'Soccer~:Dalian Transcendence vs Shanghai Shenxin L ive S treaming O nline', 1820, 1, 1467527225, 1467527225, 0, 0, 0, 0, 0, 0),
(13395, 13404, 'author', 'en', 'Soccer~:Hebei CFFC vs Shanghai SIPG L ive S treaming O nline', 1820, 1, 1467527226, 1467527226, 0, 0, 0, 0, 0, 0),
(13396, 13405, 'author', 'en', 'Soccer~:Shandong Luneng vs Guangzhou Evergrande L ive S treaming O nline', 1820, 1, 1467527231, 1467527231, 0, 0, 0, 0, 0, 0),
(13397, 13406, 'author', 'en', 'Soccer~:Shanghai Shenhua vs Changchun Yatai L ive S treaming O nline', 1820, 1, 1467527233, 1467527233, 0, 0, 0, 0, 0, 0),
(13398, 13407, 'author', 'en', 'Soccer~:Tianjin Teda vs Chongqing Lifan L ive S treaming O nline', 1820, 1, 1467527233, 1467527233, 0, 0, 0, 0, 0, 0),
(13399, 13408, 'author', 'en', 'Soccer~:Beijing Baxy vs Wuhan Zall L ive S treaming O nline', 1820, 1, 1467527234, 1467527234, 0, 0, 0, 0, 0, 0),
(13400, 13409, 'author', 'en', 'Soccer~:Beijing Renhe vs Hunan Xiangtao L ive S treaming O nline', 1820, 1, 1467527242, 1467527242, 0, 0, 0, 0, 0, 0),
(13401, 13410, 'author', 'en', 'Soccer~:Djurgardens Women vs Linköpings Women L ive S treaming O nline', 1820, 1, 1467527244, 1467527244, 0, 0, 0, 0, 0, 0),
(13402, 13411, 'author', 'en', 'Soccer~:Zhetysu vs Kairat L ive S treaming O nline', 1820, 1, 1467527244, 1467527244, 0, 0, 0, 0, 0, 0),
(13403, 13412, 'author', 'en', 'Soccer~:Umea Ik Women vs Kristianstads Women L ive S treaming O nline', 1820, 1, 1467527244, 1467527244, 0, 0, 0, 0, 0, 0),
(13404, 13413, 'author', 'en', 'Soccer~:Buriram United vs Chiangrai United L ive S treaming O nline', 1820, 1, 1467527250, 1467527250, 0, 0, 0, 0, 0, 0),
(13405, 13414, 'author', 'en', 'Soccer~:Chiangmai vs Songkhla United L ive S treaming O nline', 1820, 1, 1467527252, 1467527252, 0, 0, 0, 0, 0, 0),
(13406, 13415, 'author', 'en', 'Soccer~:FC Viktoria 1889 Berlin vs St. Pauli L ive S treaming O nline', 1820, 1, 1467527252, 1467527252, 0, 0, 0, 0, 0, 0),
(13407, 13416, 'author', 'en', 'Soccer~:Dinamo Minsk vs Slaviaa L ive S treaming O nline', 1901, 1, 1467530223, 1467530223, 0, 0, 0, 0, 0, 0),
(13408, 13417, 'author', 'en', 'Soccer~: Granit vs Belshina L ive S treaming O nline', 1901, 1, 1467530701, 1467530701, 0, 0, 0, 0, 0, 0),
(13409, 13418, 'author', 'en', 'Soccer~:Shanghai Shenhua vs Changchun Yataia L ive S treaming O nline', 1901, 1, 1467530854, 1467530854, 0, 0, 0, 0, 0, 0),
(13410, 13419, 'author', 'en', 'Purchase Tricor Delivery', 1760, 1, 1467531124, 1467531124, 0, 0, 0, 0, 0, 0),
(13411, 13420, 'author', 'en', 'Soccer~:Abengourou vs Stade d''Abidjan L ive S treaming O nline', 1901, 1, 1467531299, 1467531299, 0, 0, 0, 0, 0, 0),
(13412, 13421, 'author', 'en', 'Soccer~: Cruzeiro vs Vitória L ive S treaming O nline', 1901, 1, 1467532248, 1467532248, 0, 0, 0, 0, 0, 0),
(13413, 13422, 'author', 'en', 'Soccer~: Internacional vs Grêmio L ive S treaming O nline', 1901, 1, 1467532303, 1467532303, 0, 0, 0, 0, 0, 0),
(13414, 13423, 'author', 'en', 'Soccer~: Dragon De Yaoundé vs Botafogo L ive S treaming O nline', 1901, 1, 1467532334, 1467532334, 0, 0, 0, 0, 0, 0),
(13415, 13424, 'author', 'en', 'Soccer~: New Star vs Bamboutos L ive S treaming O nline', 1901, 1, 1467532484, 1467532484, 0, 0, 0, 0, 0, 0),
(13416, 13425, 'author', 'en', 'Soccer~: Aigle Royal vs Panthère L ive S treaming O nline', 1901, 1, 1467532516, 1467532516, 0, 0, 0, 0, 0, 0),
(13417, 13426, 'author', 'en', 'Soccer~: Cosmos De Bafia vs Les Astres L ive S treaming O nline', 1901, 1, 1467532540, 1467532540, 0, 0, 0, 0, 0, 0),
(13418, 13427, 'author', 'en', 'Soccer~: Lion Blessé vs Canon L ive S treaming O nline', 1901, 1, 1467532570, 1467532570, 0, 0, 0, 0, 0, 0),
(13419, 13428, 'author', 'en', 'Soccer~: Racing vs Eding Sport L ive S treaming O nline', 1901, 1, 1467532589, 1467532589, 0, 0, 0, 0, 0, 0),
(13420, 13429, 'author', 'en', 'Soccer~: Young Sport Academy vs Cotonsport L ive S treaming O nline', 1901, 1, 1467532611, 1467532611, 0, 0, 0, 0, 0, 0),
(13421, 13430, 'author', 'en', 'Soccer~: Apejes Academy vs Unisport Bafang L ive S treaming O nline', 1901, 1, 1467532627, 1467532627, 0, 0, 0, 0, 0, 0),
(13422, 13431, 'author', 'en', 'Soccer~: Union Douala vs Ums De Loum L ive S treaming O nline', 1901, 1, 1467532655, 1467532655, 0, 0, 0, 0, 0, 0),
(13423, 13432, 'author', 'en', 'Soccer~: Yanbian Fude vs Hangzhou L ive S treaming O nline', 1901, 1, 1467532736, 1467532736, 0, 0, 0, 0, 0, 0),
(13424, 13433, 'author', 'en', 'Soccer~: Hebei CFFC vs Shanghai SIPG L ive S treaming O nline', 1901, 1, 1467532759, 1467532759, 0, 0, 0, 0, 0, 0),
(13425, 13434, 'author', 'en', 'Soccer~: Shandong Luneng vs Guangzhou Evergrande L ive S treaming O nline', 1901, 1, 1467532787, 1467532787, 0, 0, 0, 0, 0, 0),
(13426, 13435, 'author', 'en', 'Soccer~: Dalian Transcendence vs Shanghai Shenxin L ive S treaming O nline', 1901, 1, 1467532801, 1467532801, 0, 0, 0, 0, 0, 0),
(13427, 13436, 'author', 'en', 'Soccer~: Beijing Baxy vs Wuhan Zall L ive S treaming O nline', 1901, 1, 1467532823, 1467532823, 0, 0, 0, 0, 0, 0),
(13428, 13437, 'author', 'en', 'Soccer~: Beijing Renhe vs Hunan Xiangtao L ive S treaming O nline', 1901, 1, 1467532857, 1467532857, 0, 0, 0, 0, 0, 0),
(13429, 13438, 'author', 'en', 'Soccer~: Denguélé vs Moossou L ive S treaming O nline', 1901, 1, 1467532872, 1467532872, 0, 0, 0, 0, 0, 0),
(13430, 13439, 'author', 'en', 'Soccer~: JCAT vs Africa Sports L ive S treaming O nline', 1901, 1, 1467532888, 1467532888, 0, 0, 0, 0, 0, 0),
(13431, 13440, 'author', 'en', 'Soccer~: Sporting Gagnoa vs Korhogo L ive S treaming O nline', 1901, 1, 1467532903, 1467532903, 0, 0, 0, 0, 0, 0),
(13432, 13441, 'author', 'en', 'Soccer~: El Nacional vs Emelec L ive S treaming O nline', 1901, 1, 1467532966, 1467532966, 0, 0, 0, 0, 0, 0),
(13433, 13442, 'author', 'en', 'Soccer~: LDU Quito vs Fuerza Amarilla L ive S treaming O nline', 1901, 1, 1467532985, 1467532985, 0, 0, 0, 0, 0, 0),
(13434, 13443, 'author', 'en', 'Soccer~: Mushuc Runa vs Aucas L ive S treaming O nline', 1901, 1, 1467533006, 1467533006, 0, 0, 0, 0, 0, 0),
(13435, 13444, 'author', 'en', 'Soccer~: Lahti vs KuPS L ive S treaming O nline', 1901, 1, 1467533048, 1467533048, 0, 0, 0, 0, 0, 0),
(13436, 13445, 'author', 'en', 'Soccer~: PS Kemi vs HIFK L ive S treaming O nline', 1901, 1, 1467533072, 1467533072, 0, 0, 0, 0, 0, 0),
(13437, 13446, 'author', 'en', 'Soccer~: Jaro vs Oulu L ive S treaming O nline', 1901, 1, 1467533103, 1467533103, 0, 0, 0, 0, 0, 0),
(13438, 13447, 'author', 'en', 'Soccer~: Jazz vs Ekenäs IF L ive S treaming O nline', 1901, 1, 1467533120, 1467533120, 0, 0, 0, 0, 0, 0),
(13439, 13448, 'author', 'en', 'Soccer~: JJK vs TPS L ive S treaming O nline', 1901, 1, 1467533156, 1467533156, 0, 0, 0, 0, 0, 0),
(13440, 13449, 'author', 'en', 'Soccer~:Dragon De Yaoundé vs Botafogo L ive S treaming O nline', 1820, 1, 1467533227, 1467533227, 0, 0, 0, 0, 0, 0),
(13441, 13450, 'author', 'en', 'Soccer~:New Star vs Bamboutos L ive S treaming O nline', 1820, 1, 1467533229, 1467533229, 0, 0, 0, 0, 0, 0),
(13442, 13451, 'author', 'en', 'Soccer~:Persija vs Pusamania Borneo L ive S treaming O nline', 1820, 1, 1467533230, 1467533230, 0, 0, 0, 0, 0, 0),
(13443, 13452, 'author', 'en', 'Soccer~:Ordabasy vs Atyrau L ive S treaming O nline', 1820, 1, 1467533233, 1467533233, 0, 0, 0, 0, 0, 0),
(13444, 13453, 'author', 'en', 'Soccer~:Belarus Beach vs Greece Beach L ive S treaming O nline', 1820, 1, 1467533234, 1467533234, 0, 0, 0, 0, 0, 0),
(13445, 13454, 'author', 'en', 'Soccer~:Belarus vs Greece L ive S treaming O nline', 1820, 1, 1467533234, 1467533234, 0, 0, 0, 0, 0, 0),
(13446, 13455, 'author', 'en', 'Soccer~:Viikingit vs åland L ive S treaming O nline', 1820, 1, 1467533234, 1467533234, 0, 0, 0, 0, 0, 0),
(13447, 13456, 'author', 'en', 'Soccer~:Tobol vs Irtysh L ive S treaming O nline', 1820, 1, 1467533235, 1467533235, 0, 0, 0, 0, 0, 0),
(13448, 13457, 'author', 'en', 'Soccer~:Cruzeiro vs Vitória L ive S treaming O nline', 1820, 1, 1467533238, 1467533238, 0, 0, 0, 0, 0, 0),
(13449, 13458, 'author', 'en', 'Soccer~:BBCU vs Siam Navy L ive S treaming O nline', 1820, 1, 1467533239, 1467533239, 0, 0, 0, 0, 0, 0),
(13450, 13459, 'author', 'en', 'Soccer~:Thika United vs Gor Mahia L ive S treaming O nline', 1820, 1, 1467533239, 1467533239, 0, 0, 0, 0, 0, 0),
(13451, 13460, 'author', 'en', 'Soccer~:PS TNI vs Persela L ive S treaming O nline', 1820, 1, 1467533240, 1467533240, 0, 0, 0, 0, 0, 0),
(13452, 13461, 'author', 'en', 'Soccer~:Vålerenga vs Odd L ive S treaming O nline', 1820, 1, 1467533240, 1467533240, 0, 0, 0, 0, 0, 0),
(13453, 13462, 'author', 'en', 'Soccer~:Raufoss vs Mjøndalen L ive S treaming O nline', 1820, 1, 1467533241, 1467533241, 0, 0, 0, 0, 0, 0),
(13454, 13463, 'author', 'en', 'Soccer~:Internacional vs Grêmio L ive S treaming O nline', 1820, 1, 1467533242, 1467533242, 0, 0, 0, 0, 0, 0),
(13455, 13464, 'author', 'en', 'Soccer~:France Beach vs Poland Beach L ive S treaming O nline', 1820, 1, 1467533243, 1467533243, 0, 0, 0, 0, 0, 0),
(13456, 13465, 'author', 'en', 'Soccer~:France vs Poland L ive S treaming O nline', 1820, 1, 1467533244, 1467533244, 0, 0, 0, 0, 0, 0),
(13457, 13466, 'author', 'en', 'Soccer~:Aigle Royal vs Panthère L ive S treaming O nline', 1820, 1, 1467533253, 1467533253, 0, 0, 0, 0, 0, 0),
(13458, 13467, 'author', 'en', 'Soccer~:Cosmos De Bafia vs Les Astres L ive S treaming O nline', 1820, 1, 1467533259, 1467533259, 0, 0, 0, 0, 0, 0),
(13459, 13468, 'author', 'en', 'Soccer~:Lion Blessé vs Canon L ive S treaming O nline', 1820, 1, 1467533262, 1467533262, 0, 0, 0, 0, 0, 0),
(13460, 13469, 'author', 'en', 'Soccer~:Racing vs Eding Sport L ive S treaming O nline', 1820, 1, 1467533265, 1467533265, 0, 0, 0, 0, 0, 0),
(13461, 13470, 'author', 'en', 'Soccer~:Young Sport Academy vs Cotonsport L ive S treaming O nline', 1820, 1, 1467533266, 1467533266, 0, 0, 0, 0, 0, 0),
(13462, 13471, 'author', 'en', 'Soccer~:Apejes Academy vs Unisport Bafang L ive S treaming O nline', 1820, 1, 1467533267, 1467533267, 0, 0, 0, 0, 0, 0),
(13463, 13472, 'author', 'en', 'Soccer~:Union Douala vs Ums De Loum L ive S treaming O nline', 1820, 1, 1467533268, 1467533268, 0, 0, 0, 0, 0, 0),
(13464, 13473, 'author', 'en', 'Soccer~:Bhayangkara Surabaya Utd vs Bali United L ive S treaming O nline', 1820, 1, 1467533270, 1467533270, 0, 0, 0, 0, 0, 0),
(13465, 13474, 'author', 'en', 'Soccer~:Sriwijaya vs Persipura L ive S treaming O nline', 1820, 1, 1467533280, 1467533280, 0, 0, 0, 0, 0, 0),
(13466, 13475, 'author', 'en', 'Soccer~:Dinamo Minsk vs Slavia L ive S treaming O nline', 1820, 1, 1467533283, 1467533283, 0, 0, 0, 0, 0, 0),
(13467, 13476, 'author', 'en', 'Soccer~:Aktobe vs Akzhayik L ive S treaming O nline', 1820, 1, 1467533284, 1467533284, 0, 0, 0, 0, 0, 0),
(13468, 13477, 'author', 'en', 'Soccer~:Akwa United vs Kano Pillars L ive S treaming O nline', 1820, 1, 1467533285, 1467533285, 0, 0, 0, 0, 0, 0),
(13469, 13478, 'author', 'en', 'Soccer~:Enyimba vs El Kanemi Warriors L ive S treaming O nline', 1820, 1, 1467533287, 1467533287, 0, 0, 0, 0, 0, 0),
(13470, 13479, 'author', 'en', 'Soccer~:Ifeanyi Uba vs Sunshine Stars L ive S treaming O nline', 1820, 1, 1467533287, 1467533287, 0, 0, 0, 0, 0, 0),
(13471, 13480, 'author', 'en', 'Soccer~:Lobi Stars vs Heartland L ive S treaming O nline', 1820, 1, 1467533289, 1467533289, 0, 0, 0, 0, 0, 0),
(13472, 13481, 'author', 'en', 'Soccer~:Mfm vs Ikorodu United L ive S treaming O nline', 1820, 1, 1467533301, 1467533301, 0, 0, 0, 0, 0, 0),
(13473, 13482, 'author', 'en', 'Soccer~:Niger Tornadoes vs Warri Wolves L ive S treaming O nline', 1820, 1, 1467533302, 1467533302, 0, 0, 0, 0, 0, 0),
(13474, 13483, 'author', 'en', 'Soccer~:Wikki Tourist vs Enugu Rangers L ive S treaming O nline', 1820, 1, 1467533303, 1467533303, 0, 0, 0, 0, 0, 0),
(13475, 13484, 'author', 'en', 'Soccer~:Husqvarna vs Oddevold L ive S treaming O nline', 1820, 1, 1467533304, 1467533304, 0, 0, 0, 0, 0, 0),
(13476, 13485, 'author', 'en', 'Soccer~:Mjällby vs Utsikten L ive S treaming O nline', 1820, 1, 1467533305, 1467533305, 0, 0, 0, 0, 0, 0),
(13477, 13486, 'author', 'en', 'Soccer~:Qviding FIF vs Oskarshamns AIK L ive S treaming O nline', 1820, 1, 1467533308, 1467533308, 0, 0, 0, 0, 0, 0),
(13478, 13487, 'author', 'en', 'Soccer~:Sleipner vs Vasalund L ive S treaming O nline', 1820, 1, 1467533309, 1467533309, 0, 0, 0, 0, 0, 0),
(13479, 13488, 'author', 'en', 'Soccer~:Tvååker vs Prespa Birlik L ive S treaming O nline', 1820, 1, 1467533310, 1467533310, 0, 0, 0, 0, 0, 0),
(13480, 13489, 'author', 'en', 'Soccer~:Mfm vs Ikorodu L ive S treaming O nline', 1820, 1, 1467533310, 1467533310, 0, 0, 0, 0, 0, 0),
(13481, 13490, 'author', 'en', 'Soccer~:Spain Beach vs Italy Beach L ive S treaming O nline', 1820, 1, 1467533311, 1467533311, 0, 0, 0, 0, 0, 0),
(13482, 13491, 'author', 'en', 'Soccer~:Spain vs Italy L ive S treaming O nline', 1820, 1, 1467533313, 1467533313, 0, 0, 0, 0, 0, 0),
(13483, 13492, 'author', 'en', 'Soccer~: Cosmos De Bafia vs Les Astres L ive S treaming', 1901, 1, 1467533336, 1467533336, 0, 0, 0, 0, 0, 0),
(13484, 13493, 'author', 'en', 'Soccer~: JJK vs TPS L ive S treaming ', 1901, 1, 1467533400, 1467533400, 0, 0, 0, 0, 0, 0),
(13485, 13494, 'author', 'en', 'Soccer~:Denguélé vs Moossou L ive S treaming O nline', 1820, 1, 1467534560, 1467534560, 0, 0, 0, 0, 0, 0),
(13486, 13495, 'author', 'en', 'Soccer~:JCAT vs Africa Sports L ive S treaming O nline', 1820, 1, 1467534563, 1467534563, 0, 0, 0, 0, 0, 0),
(13487, 13496, 'author', 'en', 'Soccer~:Sporting Gagnoa vs Korhogo L ive S treaming O nline', 1820, 1, 1467534564, 1467534564, 0, 0, 0, 0, 0, 0),
(13488, 13497, 'author', 'en', 'Soccer~:Lahti vs KuPS L ive S treaming O nline', 1820, 1, 1467534564, 1467534564, 0, 0, 0, 0, 0, 0),
(13489, 13498, 'author', 'en', 'Soccer~:PS Kemi vs HIFK L ive S treaming O nline', 1820, 1, 1467534565, 1467534565, 0, 0, 0, 0, 0, 0),
(13490, 13500, 'author', 'en', 'Soccer~:Jazz vs Ekenäs IF L ive S treaming O nline', 1820, 1, 1467534565, 1467534565, 0, 0, 0, 0, 0, 0),
(13491, 13499, 'author', 'en', 'Soccer~:Jaro vs Oulu L ive S treaming O nline', 1820, 1, 1467534565, 1467534565, 0, 0, 0, 0, 0, 0),
(13492, 13501, 'author', 'en', 'Soccer~:JJK vs TPS L ive S treaming O nline', 1820, 1, 1467534566, 1467534566, 0, 0, 0, 0, 0, 0),
(13493, 13502, 'author', 'en', 'Soccer~:BK-46 vs Klubi-04 L ive S treaming O nline', 1820, 1, 1467534569, 1467534569, 0, 0, 0, 0, 0, 0),
(13494, 13503, 'author', 'en', 'Soccer~:EsPa vs SalPa L ive S treaming O nline', 1820, 1, 1467534569, 1467534569, 0, 0, 0, 0, 0, 0),
(13495, 13504, 'author', 'en', 'Soccer~:GBK vs OPS L ive S treaming O nline', 1820, 1, 1467534570, 1467534570, 0, 0, 0, 0, 0, 0),
(13496, 13505, 'author', 'en', 'Soccer~:Gnistan vs Sudet L ive S treaming O nline', 1820, 1, 1467534571, 1467534571, 0, 0, 0, 0, 0, 0),
(13497, 13506, 'author', 'en', 'Soccer~:Honka vs Kiffen L ive S treaming O nline', 1820, 1, 1467534571, 1467534571, 0, 0, 0, 0, 0, 0),
(13498, 13507, 'author', 'en', 'Soccer~:Js Hercules vs ViFK L ive S treaming O nline', 1820, 1, 1467534572, 1467534572, 0, 0, 0, 0, 0, 0),
(13499, 13508, 'author', 'en', 'Soccer~:KuFu-98 vs Lahti Akatemia L ive S treaming O nline', 1820, 1, 1467534572, 1467534572, 0, 0, 0, 0, 0, 0),
(13500, 13509, 'author', 'en', 'Soccer~:MP vs Kultsu L ive S treaming O nline', 1820, 1, 1467534574, 1467534574, 0, 0, 0, 0, 0, 0),
(13501, 13510, 'author', 'en', 'Soccer~:Mps vs Vaajakoski L ive S treaming O nline', 1820, 1, 1467534574, 1467534574, 0, 0, 0, 0, 0, 0),
(13502, 13511, 'author', 'en', 'Soccer~:Pepo vs Käpa L ive S treaming O nline', 1820, 1, 1467534575, 1467534575, 0, 0, 0, 0, 0, 0),
(13503, 13512, 'author', 'en', 'Soccer~:Virkiä vs Musa L ive S treaming O nline', 1820, 1, 1467534577, 1467534577, 0, 0, 0, 0, 0, 0),
(13504, 13513, 'author', 'en', 'Soccer~:Sturm Graz vs Celtic L ive S treaming O nline', 1820, 1, 1467534579, 1467534579, 0, 0, 0, 0, 0, 0),
(13505, 13514, 'author', 'en', 'Soccer~:Granit vs Belshina L ive S treaming O nline', 1820, 1, 1467534579, 1467534579, 0, 0, 0, 0, 0, 0),
(13506, 13515, 'author', 'en', 'Soccer~:Molde vs Viking L ive S treaming O nline', 1820, 1, 1467534580, 1467534580, 0, 0, 0, 0, 0, 0),
(13507, 13516, 'author', 'en', 'Soccer~:Sarpsborg 08 vs Brann L ive S treaming O nline', 1820, 1, 1467534582, 1467534582, 0, 0, 0, 0, 0, 0),
(13508, 13517, 'author', 'en', 'Soccer~:Stabæk vs Sogndal L ive S treaming O nline', 1820, 1, 1467534584, 1467534584, 0, 0, 0, 0, 0, 0),
(13509, 13518, 'author', 'en', 'Soccer~:Bryne vs Strømmen L ive S treaming O nline', 1820, 1, 1467534584, 1467534584, 0, 0, 0, 0, 0, 0),
(13510, 13519, 'author', 'en', 'Soccer~:Hødd vs Ranheim L ive S treaming O nline', 1820, 1, 1467534585, 1467534585, 0, 0, 0, 0, 0, 0),
(13511, 13520, 'author', 'en', 'Soccer~:Jerv vs Kongsvinger L ive S treaming O nline', 1820, 1, 1467534587, 1467534587, 0, 0, 0, 0, 0, 0),
(13512, 13521, 'author', 'en', 'Soccer~:Sandefjord vs Levanger L ive S treaming O nline', 1820, 1, 1467534589, 1467534589, 0, 0, 0, 0, 0, 0),
(13513, 13522, 'author', 'en', 'Soccer~:Ullensaker / Kisa vs KFUM L ive S treaming O nline', 1820, 1, 1467534589, 1467534589, 0, 0, 0, 0, 0, 0),
(13514, 13523, 'author', 'en', 'Soccer~:Åsane vs Kristiansund L ive S treaming O nline', 1820, 1, 1467534589, 1467534589, 0, 0, 0, 0, 0, 0),
(13515, 13524, 'author', 'en', 'Soccer~:Follo vs Skeid L ive S treaming O nline', 1820, 1, 1467534592, 1467534592, 0, 0, 0, 0, 0, 0),
(13516, 13525, 'author', 'en', 'Soccer~:Defensor La Bocana vs Melgar L ive S treaming O nline', 1820, 1, 1467534594, 1467534594, 0, 0, 0, 0, 0, 0),
(13517, 13526, 'author', 'en', 'Soccer~:Assyriska vs Sirius L ive S treaming O nline', 1820, 1, 1467534595, 1467534595, 0, 0, 0, 0, 0, 0),
(13518, 13527, 'author', 'en', 'Soccer~:Norrby vs Höllviken L ive S treaming O nline', 1820, 1, 1467534596, 1467534596, 0, 0, 0, 0, 0, 0),
(13519, 13528, 'author', 'en', 'Soccer~:New York City vs New York RB L ive S treaming O nline', 1820, 1, 1467534598, 1467534598, 0, 0, 0, 0, 0, 0),
(13520, 13529, 'author', 'en', 'Soccer~:Dynamo Kyiv vs Ludogorets L ive S treaming O nline', 1820, 1, 1467534600, 1467534600, 0, 0, 0, 0, 0, 0),
(13521, 13530, 'author', 'en', 'Soccer~:El Nacional vs Emelec L ive S treaming O nline', 1820, 1, 1467534603, 1467534603, 0, 0, 0, 0, 0, 0),
(13522, 13531, 'author', 'en', 'Soccer~:Russia Beach vs Switzerland Beach L ive S treaming O nline', 1820, 1, 1467534603, 1467534603, 0, 0, 0, 0, 0, 0),
(13523, 13532, 'author', 'en', 'Soccer~:Russia vs Switzerland L ive S treaming O nline', 1820, 1, 1467534606, 1467534606, 0, 0, 0, 0, 0, 0),
(13524, 13533, 'author', 'en', 'Soccer~:LDU Quito vs Fuerza Amarilla L ive S treaming O nline', 1820, 1, 1467534606, 1467534606, 0, 0, 0, 0, 0, 0),
(13525, 13534, 'author', 'en', 'Soccer~:Mushuc Runa vs Aucas L ive S treaming O nline', 1820, 1, 1467534606, 1467534606, 0, 0, 0, 0, 0, 0),
(13526, 13535, 'author', 'en', 'Soccer~:Ayacucho FC vs Real Garcilaso L ive S treaming O nline', 1820, 1, 1467534608, 1467534608, 0, 0, 0, 0, 0, 0),
(13527, 13536, 'author', 'en', 'Soccer~:Abengourou vs Stade d''Abidjan L ive S treaming W atch O nline', 1820, 1, 1467534976, 1467534976, 0, 0, 0, 0, 0, 0),
(13528, 13537, 'author', 'en', 'Soccer~: Viikingit vs åland L ive S treaming O nline', 1901, 1, 1467535714, 1467535714, 0, 0, 0, 0, 0, 0),
(13529, 13538, 'author', 'en', 'Soccer~: BK-46 vs Klubi-04 L ive S treaming O nline', 1901, 1, 1467535758, 1467535758, 0, 0, 0, 0, 0, 0),
(13530, 13539, 'author', 'en', 'Soccer~: EsPa vs SalPa L ive S treaming O nline', 1901, 1, 1467535776, 1467535776, 0, 0, 0, 0, 0, 0),
(13531, 13540, 'author', 'en', 'Soccer~: GBK vs OPS L ive S treaming O nline', 1901, 1, 1467535811, 1467535811, 0, 0, 0, 0, 0, 0),
(13532, 13541, 'author', 'en', 'Soccer~: Gnistan vs Sudet L ive S treaming O nline', 1901, 1, 1467535829, 1467535829, 0, 0, 0, 0, 0, 0),
(13533, 13542, 'author', 'en', 'Soccer~: Honka vs Kiffen L ive S treaming O nline', 1901, 1, 1467535856, 1467535856, 0, 0, 0, 0, 0, 0),
(13534, 13543, 'author', 'en', 'Soccer~: Js Hercules vs ViFK L ive S treaming O nline', 1901, 1, 1467535901, 1467535901, 0, 0, 0, 0, 0, 0),
(13535, 13544, 'author', 'en', 'Soccer~: KuFu-98 vs Lahti Akatemia L ive S treaming O nline', 1901, 1, 1467535948, 1467535948, 0, 0, 0, 0, 0, 0),
(13536, 13545, 'author', 'en', 'Soccer~: MP vs Kultsu L ive S treaming O nline', 1901, 1, 1467535976, 1467535976, 0, 0, 0, 0, 0, 0),
(13537, 13546, 'author', 'en', 'Soccer~: Mps vs Vaajakoski L ive S treaming O nline', 1901, 1, 1467536008, 1467536008, 0, 0, 0, 0, 0, 0),
(13538, 13547, 'author', 'en', 'Soccer~: Pepo vs Käpa L ive S treaming O nline', 1901, 1, 1467536056, 1467536056, 0, 0, 0, 0, 0, 0),
(13539, 13548, 'author', 'en', 'Soccer~: Virkiä vs Musa L ive S treaming O nline', 1901, 1, 1467536075, 1467536075, 0, 0, 0, 0, 0, 0),
(13540, 13549, 'author', 'en', 'Soccer~: Persija vs Pusamania Borneo L ive S treaming O nline', 1901, 1, 1467536140, 1467536140, 0, 0, 0, 0, 0, 0),
(13541, 13550, 'author', 'en', 'Soccer~: PS TNI vs Persela L ive S treaming O nline', 1901, 1, 1467536203, 1467536203, 0, 0, 0, 0, 0, 0),
(13542, 13551, 'author', 'en', 'Soccer~: Bhayangkara Surabaya Utd vs Bali United L ive S treaming O nline', 1901, 1, 1467536235, 1467536235, 0, 0, 0, 0, 0, 0),
(13543, 13552, 'author', 'en', 'Soccer~: Sriwijaya vs Persipura L ive S treaming O nline', 1901, 1, 1467536288, 1467536288, 0, 0, 0, 0, 0, 0),
(13544, 13553, 'author', 'en', 'Soccer~: Fagiano Okayama vs Shimizu S-Pulse L ive S treaming O nline', 1901, 1, 1467536321, 1467536321, 0, 0, 0, 0, 0, 0),
(13545, 13554, 'author', 'en', 'Soccer~: Giravanz Kitakyushu vs Tokyo Verdy L ive S treaming O nline', 1901, 1, 1467536337, 1467536337, 0, 0, 0, 0, 0, 0),
(13546, 13555, 'author', 'en', 'Soccer~: Mito Hollyhock vs Matsumoto Yamaga L ive S treaming O nline', 1901, 1, 1467536357, 1467536357, 0, 0, 0, 0, 0, 0),
(13547, 13556, 'author', 'en', 'Soccer~: Montedio Yamagata vs Renofa Yamaguchi L ive S treaming O nline', 1901, 1, 1467536430, 1467536430, 0, 0, 0, 0, 0, 0),
(13548, 13557, 'author', 'en', 'Soccer~: Roasso Kumamoto vs Cerezo Osaka L ive S treaming O nline', 1901, 1, 1467536494, 1467536494, 0, 0, 0, 0, 0, 0),
(13549, 13558, 'author', 'en', 'Soccer~: Tokushima Vortis vs V-Varen Nagasaki L ive S treaming O nline', 1901, 1, 1467536527, 1467536527, 0, 0, 0, 0, 0, 0),
(13550, 13559, 'author', 'en', 'Soccer~: Zweigen Kanazawa vs Kamatamare Sanuki L ive S treaming O nline', 1901, 1, 1467536557, 1467536557, 0, 0, 0, 0, 0, 0),
(13551, 13560, 'author', 'en', 'Soccer~: Ehime vs JEF United L ive S treaming O nline', 1901, 1, 1467536593, 1467536593, 0, 0, 0, 0, 0, 0),
(13552, 13561, 'author', 'en', 'Soccer~: Gifu vs Kyoto Sanga L ive S treaming O nline', 1901, 1, 1467536627, 1467536627, 0, 0, 0, 0, 0, 0),
(13553, 13562, 'author', 'en', 'Soccer~: ThespaKusatsu Gunma vs Machida Zelvia L ive S treaming O nline', 1901, 1, 1467536657, 1467536657, 0, 0, 0, 0, 0, 0),
(13554, 13563, 'author', 'en', 'Soccer~: Aktobe vs Akzhayik L ive S treaming O nline', 1901, 1, 1467536700, 1467536700, 0, 0, 0, 0, 0, 0),
(13555, 13564, 'author', 'en', 'Soccer~: Ordabasy vs Atyrau L ive S treaming O nline', 1901, 1, 1467536725, 1467536725, 0, 0, 0, 0, 0, 0),
(13556, 13565, 'author', 'en', 'Soccer~: Zhetysu vs Kairat L ive S treaming O nline', 1901, 1, 1467536751, 1467536751, 0, 0, 0, 0, 0, 0),
(13557, 13566, 'author', 'en', 'Soccer~: KRA vs SoNy Sugar L ive S treaming O nline', 1901, 1, 1467536868, 1467536868, 0, 0, 0, 0, 0, 0),
(13558, 13567, 'author', 'en', 'Soccer~: Thika United vs Gor Mahia L ive S treaming O nline', 1901, 1, 1467536907, 1467536907, 0, 0, 0, 0, 0, 0),
(13559, 13568, 'author', 'en', 'Soccer~: Incheon United vs Jeju United L ive S treaming O nline', 1901, 1, 1467536925, 1467536925, 0, 0, 0, 0, 0, 0),
(13560, 13569, 'author', 'en', 'Soccer~: Gwangju vs Pohang Steelers L ive S treaming O nline', 1901, 1, 1467536957, 1467536957, 0, 0, 0, 0, 0, 0),
(13561, 13570, 'author', 'en', 'Soccer~: Suwon City vs Jeonbuk Motors L ive S treaming O nline', 1901, 1, 1467537045, 1467537045, 0, 0, 0, 0, 0, 0),
(13562, 13571, 'author', 'en', 'Soccer~: Akwa United vs Kano Pillars L ive S treaming O nline', 1901, 1, 1467537064, 1467537064, 0, 0, 0, 0, 0, 0),
(13563, 13572, 'author', 'en', 'Soccer~: Enyimba vs El Kanemi Warriors L ive S treaming O nline', 1901, 1, 1467537226, 1467537226, 0, 0, 0, 0, 0, 0),
(13564, 13573, 'author', 'en', 'Soccer~: Ifeanyi Uba vs Sunshine Stars L ive S treaming O nline', 1901, 1, 1467537268, 1467537268, 0, 0, 0, 0, 0, 0),
(13565, 13574, 'author', 'en', 'Soccer~: Lobi Stars vs Heartland L ive S treaming O nline', 1901, 1, 1467537296, 1467537296, 0, 0, 0, 0, 0, 0),
(13566, 13575, 'author', 'en', 'Soccer~: Mfm vs Ikorodu United L ive S treaming O nline', 1901, 1, 1467537334, 1467537334, 0, 0, 0, 0, 0, 0),
(13567, 13576, 'author', 'en', 'Soccer~: Niger Tornadoes vs Warri Wolves L ive S treaming O nline', 1901, 1, 1467537392, 1467537392, 0, 0, 0, 0, 0, 0),
(13568, 13577, 'author', 'en', 'Soccer~: Wikki Tourist vs Enugu Rangers L ive S treaming O nline', 1901, 1, 1467537416, 1467537416, 0, 0, 0, 0, 0, 0),
(13569, 13578, 'author', 'en', 'Soccer~: Molde vs Viking L ive S treaming O nline', 1901, 1, 1467537445, 1467537445, 0, 0, 0, 0, 0, 0),
(13570, 13579, 'author', 'en', 'Soccer~: Sarpsborg 08 vs Brann L ive S treaming O nline', 1901, 1, 1467537462, 1467537462, 0, 0, 0, 0, 0, 0),
(13571, 13580, 'author', 'en', 'Soccer~: Stabæk vs Sogndal L ive S treaming O nline', 1901, 1, 1467537490, 1467537490, 0, 0, 0, 0, 0, 0),
(13572, 13581, 'author', 'en', 'Soccer~: Raufoss vs Mjøndalen L ive S treaming O nline', 1901, 1, 1467537514, 1467537514, 0, 0, 0, 0, 0, 0),
(13573, 13582, 'author', 'en', 'Soccer~: Bryne vs Strømmen L ive S treaming O nline', 1901, 1, 1467537546, 1467537546, 0, 0, 0, 0, 0, 0),
(13574, 13583, 'author', 'en', 'Soccer~: Hødd vs Ranheim L ive S treaming O nline', 1901, 1, 1467537587, 1467537587, 0, 0, 0, 0, 0, 0),
(13575, 13584, 'author', 'en', 'Soccer~: Jerv vs Kongsvinger L ive S treaming O nline', 1901, 1, 1467537605, 1467537605, 0, 0, 0, 0, 0, 0),
(13576, 13585, 'author', 'en', 'Soccer~: Sandefjord vs Levanger L ive S treaming O nline', 1901, 1, 1467537625, 1467537625, 0, 0, 0, 0, 0, 0),
(13577, 13586, 'author', 'en', 'Soccer~: Ullensaker / Kisa vs KFUM L ive S treaming O nline', 1901, 1, 1467537645, 1467537645, 0, 0, 0, 0, 0, 0),
(13578, 13587, 'author', 'en', 'Soccer~: Åsane vs Kristiansund L ive S treaming O nline', 1901, 1, 1467537665, 1467537665, 0, 0, 0, 0, 0, 0),
(13579, 13588, 'author', 'en', 'Soccer~: Follo vs Skeid L ive S treaming O nline', 1901, 1, 1467537684, 1467537684, 0, 0, 0, 0, 0, 0),
(13580, 13589, 'author', 'en', 'Soccer~: Defensor La Bocana vs Melgar L ive S treaming O nline', 1901, 1, 1467537708, 1467537708, 0, 0, 0, 0, 0, 0),
(13581, 13590, 'author', 'en', 'Soccer~: Stallions vs PSG L ive S treaming O nline', 1901, 1, 1467537745, 1467537745, 0, 0, 0, 0, 0, 0),
(13582, 13591, 'author', 'en', 'Soccer~: Djurgardens Women vs Linköpings Women L ive S treaming O nline', 1901, 1, 1467537778, 1467537778, 0, 0, 0, 0, 0, 0),
(13583, 13592, 'author', 'en', 'Soccer~: Umea Ik Women vs Kristianstads Women L ive S treaming O nline', 1901, 1, 1467537812, 1467537812, 0, 0, 0, 0, 0, 0),
(13584, 13593, 'author', 'en', 'Soccer~: Assyriska vs Sirius L ive S treaming O nline', 1901, 1, 1467537839, 1467537839, 0, 0, 0, 0, 0, 0),
(13585, 13594, 'author', 'en', 'Soccer~: Husqvarna vs Oddevold L ive S treaming O nline', 1901, 1, 1467537857, 1467537857, 0, 0, 0, 0, 0, 0),
(13586, 13595, 'author', 'en', 'Soccer~: Mjällby vs Utsikten L ive S treaming O nline', 1901, 1, 1467537886, 1467537886, 0, 0, 0, 0, 0, 0),
(13587, 13596, 'author', 'en', 'Soccer~: Qviding FIF vs Oskarshamns AIK L ive S treaming O nline', 1901, 1, 1467537944, 1467537944, 0, 0, 0, 0, 0, 0),
(13588, 13597, 'author', 'en', 'Soccer~: Sleipner vs Vasalund L ive S treaming O nline', 1901, 1, 1467537994, 1467537994, 0, 0, 0, 0, 0, 0),
(13589, 13598, 'author', 'en', 'Soccer~: Tvååker vs Prespa Birlik L ive S treaming O nline', 1901, 1, 1467538036, 1467538036, 0, 0, 0, 0, 0, 0),
(13590, 13599, 'author', 'en', 'Soccer~: Norrby vs Höllviken L ive S treaming O nline', 1901, 1, 1467538075, 1467538075, 0, 0, 0, 0, 0, 0),
(13591, 13600, 'author', 'en', 'Soccer~: Chainat Hornbill vs Sisaket L ive S treaming O nline', 1901, 1, 1467538167, 1467538167, 0, 0, 0, 0, 0, 0),
(13592, 13601, 'author', 'en', 'Soccer~: Sukhothai vs Chonburi L ive S treaming O nline', 1901, 1, 1467538181, 1467538181, 0, 0, 0, 0, 0, 0),
(13593, 13602, 'author', 'en', 'Soccer~: Buriram United vs Chiangrai United L ive S treaming O nline', 1901, 1, 1467538198, 1467538198, 0, 0, 0, 0, 0, 0),
(13594, 13603, 'author', 'en', 'Soccer~: BBCU vs Siam Navy L ive S treaming O nline', 1901, 1, 1467538220, 1467538220, 0, 0, 0, 0, 0, 0),
(13595, 13604, 'author', 'en', 'Soccer~: Air Force Central vs Rayong L ive S treaming O nline', 1901, 1, 1467538240, 1467538240, 0, 0, 0, 0, 0, 0),
(13596, 13605, 'author', 'en', 'Soccer~: Lampang vs Krabi L ive S treaming O nline', 1901, 1, 1467538257, 1467538257, 0, 0, 0, 0, 0, 0),
(13597, 13606, 'author', 'en', 'Soccer~: Thai Honda vs Samut Songkhram L ive S treaming O nline', 1901, 1, 1467538275, 1467538275, 0, 0, 0, 0, 0, 0),
(13598, 13607, 'author', 'en', 'Soccer~: Chiangmai vs Songkhla United L ive S treaming O nline', 1901, 1, 1467538290, 1467538290, 0, 0, 0, 0, 0, 0),
(13599, 13608, 'author', 'en', 'Soccer~: New York City vs New York RB L ive S treaming O nline', 1901, 1, 1467538309, 1467538309, 0, 0, 0, 0, 0, 0),
(13600, 13609, 'author', 'en', 'Soccer~: Mfm vs Ikorodu L ive S treaming O nline', 1901, 1, 1467538330, 1467538330, 0, 0, 0, 0, 0, 0),
(13601, 13610, 'author', 'en', 'Soccer~: Da Nang vs Flc Thanh Hoa L ive S treaming O nline', 1901, 1, 1467538362, 1467538362, 0, 0, 0, 0, 0, 0),
(13602, 13611, 'author', 'en', 'Soccer~: Quang Nam vs Ha Noi L ive S treaming O nline', 1901, 1, 1467538377, 1467538377, 0, 0, 0, 0, 0, 0),
(13603, 13612, 'author', 'en', 'Soccer~: Can Tho vs Hoang Anh Gia Lai L ive S treaming O nline', 1901, 1, 1467538396, 1467538396, 0, 0, 0, 0, 0, 0),
(13604, 13613, 'author', 'en', 'Soccer~: Hai Phong vs Dong Thap L ive S treaming O nline', 1901, 1, 1467538416, 1467538416, 0, 0, 0, 0, 0, 0),
(13605, 13614, 'author', 'en', 'Soccer~: Sanna Khanh Hoa vs Binh Duong L ive S treaming O nline', 1901, 1, 1467538433, 1467538433, 0, 0, 0, 0, 0, 0),
(13606, 13615, 'author', 'en', 'Soccer~: Song Lam Nghe An vs Quang Ninh L ive S treaming O nline', 1901, 1, 1467538451, 1467538451, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(24668, 24677, 'author', 'en', '[YouTube] Scream Season 2 Episode 6 "Jeepers Creepers" Online', 1949, 1, 1467694962, 1467695066, 0, 0, 0, 0, 0, 0),
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(24765, 24774, 'author', 'en', '[Live~HD] Ordabasy vs Čukarički Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722476, 1467722476, 0, 0, 0, 0, 0, 0),
(24766, 24775, 'author', 'en', '[Live~HD] Shirak vs Dila Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722484, 1467722484, 0, 0, 0, 0, 0, 0),
(24767, 24776, 'author', 'en', '[Live~HD] Kairat vs Teuta Durrës Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722493, 1467722493, 0, 0, 0, 0, 0, 0),
(24768, 24777, 'author', 'en', '[Live~HD] Spartak Myjava vs Admira Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722501, 1467722501, 0, 0, 0, 0, 0, 0),
(24769, 24778, 'author', 'en', '[Live~HD] Dacia vs Kapaz Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722510, 1467722510, 0, 0, 0, 0, 0, 0),
(24770, 24779, 'author', 'en', '[Live~HD] Pyunik vs Europa Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722520, 1467722520, 0, 0, 0, 0, 0, 0),
(24771, 24780, 'author', 'en', '[Live~HD] Samtredia vs Qabala Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722525, 1467722525, 0, 0, 0, 0, 0, 0),
(24772, 24781, 'author', 'en', '[Live~HD] Rudar vs Kukësi Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722533, 1467722533, 0, 0, 0, 0, 0, 0),
(24773, 24782, 'author', 'en', '[Live~HD] Jelgava vs Breidablik Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722541, 1467722541, 0, 0, 0, 0, 0, 0),
(24774, 24783, 'author', 'en', '[Live~HD] Nõmme Kalju vs Trakai Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722549, 1467722549, 0, 0, 0, 0, 0, 0),
(24775, 24784, 'author', 'en', '[Live~HD] MTK vs Aktobe Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722560, 1467722560, 0, 0, 0, 0, 0, 0),
(24776, 24785, 'author', 'en', '[Live~HD] Lokomotiva Zagreb vs UE Santa Coloma Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722576, 1467722576, 0, 0, 0, 0, 0, 0),
(24777, 24786, 'author', 'en', '[Live~HD] HJK vs Atlantas Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722586, 1467722586, 0, 0, 0, 0, 0, 0),
(24778, 24787, 'author', 'en', '[Live~HD] Neftçi vs Balzan Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722594, 1467722594, 0, 0, 0, 0, 0, 0),
(24779, 24788, 'author', 'en', '[Live~HD] Spartaks vs Dinamo Minsk Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722601, 1467722601, 0, 0, 0, 0, 0, 0),
(24780, 24789, 'author', 'en', '[Live~HD] Zagłębie Lubin vs Slavia Sofia Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722609, 1467722609, 0, 0, 0, 0, 0, 0),
(24781, 24790, 'author', 'en', '[Live~HD] Mariehamn vs Odd Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722617, 1467722617, 0, 0, 0, 0, 0, 0),
(24782, 24791, 'author', 'en', '[Live~HD] Radnik Bijeljina vs Beroe Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722626, 1467722626, 0, 0, 0, 0, 0, 0),
(24783, 24792, 'author', 'en', '[Live~HD] Debrecen vs La Fiorita Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722635, 1467722635, 0, 0, 0, 0, 0, 0),
(24784, 24793, 'author', 'en', '[Live~HD] RoPS vs Shamrock Rovers Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722643, 1467722643, 0, 0, 0, 0, 0, 0),
(24785, 24794, 'author', 'en', '[Live~HD] Omonia Nicosia vs Banants Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722651, 1467722651, 0, 0, 0, 0, 0, 0),
(24786, 24795, 'author', 'en', '[Live~HD] Fola Esch vs Aberdeen Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722659, 1467722659, 0, 0, 0, 0, 0, 0),
(24787, 24796, 'author', 'en', '[Live~HD] Brøndby vs Valur Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722674, 1467722674, 0, 0, 0, 0, 0, 0),
(24788, 24797, 'author', 'en', '[Live~HD] Beitar Jerusalem vs Sloboda Tuzla Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722711, 1467722711, 0, 0, 0, 0, 0, 0),
(24789, 24798, 'author', 'en', '[Live~HD] Gorica vs Maccabi Tel Aviv Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722720, 1467722720, 0, 0, 0, 0, 0, 0),
(24790, 24799, 'author', 'en', '[Live~HD] Budućnost vs Rabotnički Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722737, 1467722737, 0, 0, 0, 0, 0, 0),
(24791, 24800, 'author', 'en', '[Live~HD] Slovan Bratislava vs Partizani Tirana Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722747, 1467722747, 0, 0, 0, 0, 0, 0),
(24792, 24801, 'author', 'en', '[Live~HD] HB vs Levadia Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722756, 1467722756, 0, 0, 0, 0, 0, 0),
(24793, 24802, 'author', 'en', '[Live~HD] Zaria vs Videoton Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722766, 1467722766, 0, 0, 0, 0, 0, 0),
(24794, 24803, 'author', 'en', '[Live~HD] Sūduva vs Midtjylland Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722775, 1467722775, 0, 0, 0, 0, 0, 0),
(24795, 24804, 'author', 'en', '[Live~HD] Stabæk vs Connah''s Quay Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722840, 1467722840, 0, 0, 0, 0, 0, 0),
(24796, 24805, 'author', 'en', '[Live~HD] Bala Town vs AIK Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722869, 1467722869, 0, 0, 0, 0, 0, 0),
(24797, 24806, 'author', 'en', '[Live~HD] Víkingur vs Ventspils Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467722945, 1467722945, 0, 0, 0, 0, 0, 0),
(24798, 24807, 'author', 'en', '[Live~HD] Vojvodina vs Bokelj Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723032, 1467723032, 0, 0, 0, 0, 0, 0),
(24799, 24808, 'author', 'en', '[Live~HD] Llandudno vs IFK Göteborg Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723100, 1467723100, 0, 0, 0, 0, 0, 0),
(24800, 24809, 'author', 'en', '[Live~HD] Sileks vs Vaduz Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723133, 1467723133, 0, 0, 0, 0, 0, 0),
(24801, 24810, 'author', 'en', '[Live~HD] Shakhtyor vs Nsí Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723154, 1467723154, 0, 0, 0, 0, 0, 0),
(24802, 24811, 'author', 'en', '[Live~HD] Hibernians vs Spartak Trnava Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723172, 1467723172, 0, 0, 0, 0, 0, 0),
(24803, 24812, 'author', 'en', '[Live~HD] Cracovia Kraków vs Škendija 79 Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723195, 1467723195, 0, 0, 0, 0, 0, 0),
(24804, 24813, 'author', 'en', '[Live~HD] Isloch vs BATE Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723216, 1467723216, 0, 0, 0, 0, 0, 0),
(24805, 24814, 'author', 'en', '[Live~HD] ASEC vs Denguélé Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723237, 1467723237, 0, 0, 0, 0, 0, 0),
(24806, 24815, 'author', 'en', '[Live~HD] Laos vs Ceres Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723254, 1467723254, 0, 0, 0, 0, 0, 0),
(24807, 24816, 'author', 'en', '[Live~HD] Guangzhou Evergrande vs Schalke 04 Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723288, 1467723288, 0, 0, 0, 0, 0, 0),
(24808, 24817, 'author', 'en', '[Live~HD] 1860 München vs Austria Wien Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723308, 1467723308, 0, 0, 0, 0, 0, 0),
(24809, 24818, 'author', 'en', '[Live~HD] Panathinaikos vs Ankaraspor Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723333, 1467723333, 0, 0, 0, 0, 0, 0),
(24810, 24819, 'author', 'en', '[Live~HD] Germany vs France Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723378, 1467723378, 0, 0, 0, 0, 0, 0),
(24811, 24820, 'author', 'en', 'FULL-ESSES Wentworth Season 4 Episode 9 Online s4e9 Free', 1967, 1, 1467723521, 1467723521, 0, 0, 0, 0, 0, 0),
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(24813, 24822, 'author', 'en', '[Live~HD] Lusitanos vs Domžale Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723761, 1467723761, 0, 0, 0, 0, 0, 0),
(24814, 24823, 'author', 'en', '[Live~HD] Cliftonville vs Differdange 03 Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723784, 1467723784, 0, 0, 0, 0, 0, 0),
(24815, 24824, 'author', 'en', '[Live~HD] Cork City vs Linfield Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723819, 1467723819, 0, 0, 0, 0, 0, 0),
(24816, 24825, 'author', 'en', '[Live~HD] Glenavon vs KR Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723849, 1467723849, 0, 0, 0, 0, 0, 0),
(24817, 24826, 'author', 'en', '[Live~HD] América Mineiro vs Fortaleza Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467723876, 1467723876, 0, 0, 0, 0, 0, 0),
(24818, 24827, 'author', 'en', '[Live~HD] General Díaz vs General Caballero Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724041, 1467724041, 0, 0, 0, 0, 0, 0),
(24819, 24828, 'author', 'en', '[Live~HD] Guaraní vs Rubio Ñú Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724069, 1467724069, 0, 0, 0, 0, 0, 0),
(24820, 24829, 'author', 'en', '[Live~HD] Libertad vs River Plate Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724086, 1467724086, 0, 0, 0, 0, 0, 0),
(24821, 24830, 'author', 'en', '[Live~HD] Nacional Asunción vs Deportivo Capiatá Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724110, 1467724110, 0, 0, 0, 0, 0, 0),
(24822, 24831, 'author', 'en', '[Live~HD] Sol de América vs Cerro Porteño Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724126, 1467724126, 0, 0, 0, 0, 0, 0),
(24823, 24832, 'author', 'en', 'HD-Special "Wentworth Season 4 Episode 9" On-line s4e9 Free', 1967, 1, 1467724234, 1467724234, 0, 0, 0, 0, 0, 0),
(24824, 24833, 'author', 'en', '[Live~HD] Sportivo Luqueño vs Olimpia Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724373, 1467724373, 0, 0, 0, 0, 0, 0),
(24825, 24834, 'author', 'en', '[Live~HD] Comerciantes Unidos vs Universitario Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724394, 1467724394, 0, 0, 0, 0, 0, 0),
(24826, 24835, 'author', 'en', '[Live~HD] Independiente del Valle vs Boca Juniors Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724416, 1467724416, 0, 0, 0, 0, 0, 0),
(24827, 24836, 'author', 'en', '[Live~HD] Portland Timbers II vs Colorado Springs Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724466, 1467724466, 0, 0, 0, 0, 0, 0),
(24828, 24837, 'author', 'en', '[Live~HD] Atlético vs Orsomarso Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467724541, 1467724541, 0, 0, 0, 0, 0, 0),
(24829, 24838, 'author', 'en', '[Live~HD] Deportes Quindío vs Deportivo Pereira Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725142, 1467725142, 0, 0, 0, 0, 0, 0),
(24830, 24839, 'author', 'en', '[Live~HD] Expreso Rojo vs Barranquilla Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725155, 1467725155, 0, 0, 0, 0, 0, 0),
(24831, 24840, 'author', 'en', '[Live~HD] Llaneros vs Valledupar Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725239, 1467725239, 0, 0, 0, 0, 0, 0),
(24832, 24841, 'author', 'en', '[Live~HD] Real Cartagena vs Bogotá Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725260, 1467725260, 0, 0, 0, 0, 0, 0),
(24833, 24842, 'author', 'en', '[Live~HD] Real Santander vs América de Cali Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725276, 1467725276, 0, 0, 0, 0, 0, 0),
(24834, 24843, 'author', 'en', '[Live~HD] Universitario Popayán vs Cúcuta Deportivo Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725291, 1467725291, 0, 0, 0, 0, 0, 0),
(24835, 24844, 'author', 'en', '[Live~HD] Unión Magdalena vs Leones Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725311, 1467725311, 0, 0, 0, 0, 0, 0),
(24836, 24845, 'author', 'en', 'Buy Flomax D Online', 1888, 1, 1467725318, 1467725318, 0, 0, 0, 0, 0, 0),
(24837, 24846, 'author', 'en', '[Live~HD] JCAT vs AFAD Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725335, 1467725335, 0, 0, 0, 0, 0, 0),
(24838, 24847, 'author', 'en', '[Live~HD] Korhogo vs SOA Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725353, 1467725353, 0, 0, 0, 0, 0, 0),
(24839, 24848, 'author', 'en', '[Live~HD] Moossou vs Yopougon Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725536, 1467725536, 0, 0, 0, 0, 0, 0),
(24840, 24849, 'author', 'en', '[Live~HD] Sporting Gagnoa vs Denguélé Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725586, 1467725586, 0, 0, 0, 0, 0, 0),
(24841, 24850, 'author', 'en', '[Live~HD] Stade d''Abidjan vs Africa Sports Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725603, 1467725603, 0, 0, 0, 0, 0, 0),
(24842, 24851, 'author', 'en', '[Live~HD] Séwé Sport vs ASEC Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725628, 1467725628, 0, 0, 0, 0, 0, 0),
(24843, 24852, 'author', 'en', '[Live~HD] Tanda vs Abengourou Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725648, 1467725648, 0, 0, 0, 0, 0, 0),
(24844, 24853, 'author', 'en', '[Live~HD] 1º de Agosto vs Académica do Lobito Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725668, 1467725668, 0, 0, 0, 0, 0, 0),
(24845, 24854, 'author', 'en', '[Live~HD] 1º de Maio vs Desportivo Huíla Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725776, 1467725776, 0, 0, 0, 0, 0, 0),
(24846, 24855, 'author', 'en', '[Live~HD] ASA vs Recreativo do Libolo Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725856, 1467725856, 0, 0, 0, 0, 0, 0),
(24847, 24856, 'author', 'en', '[Live~HD] Benfica Luanda vs Progresso Lunda Sul Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725924, 1467725924, 0, 0, 0, 0, 0, 0),
(24848, 24857, 'author', 'en', '[Live~HD] Interclube vs Sagrada Esperança Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467725979, 1467725979, 0, 0, 0, 0, 0, 0),
(24849, 24858, 'author', 'en', '[Live~HD] Kabuscorp vs Porcelana Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726002, 1467726002, 0, 0, 0, 0, 0, 0),
(24850, 24859, 'author', 'en', '[Live~HD] Petro de Luanda vs Recreativo da Caála Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726017, 1467726017, 0, 0, 0, 0, 0, 0),
(24851, 24860, 'author', 'en', '[Live~HD] Progresso vs 4 De Abril Do Cuando Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726036, 1467726036, 0, 0, 0, 0, 0, 0),
(24852, 24861, 'author', 'en', '[Live~HD] Qarabağ vs Rapid Wien Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726055, 1467726055, 0, 0, 0, 0, 0, 0),
(24853, 24862, 'author', 'en', '[Live~HD] Liaoning Whowin vs Shijiazhuang Ever Bright Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726071, 1467726071, 0, 0, 0, 0, 0, 0),
(24854, 24863, 'author', 'en', '[Live~HD] Al Ittihad vs Wadi Degla Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726089, 1467726089, 0, 0, 0, 0, 0, 0),
(24855, 24864, 'author', 'en', '[Live~HD] Ghazl El Mehalla vs Al Masry Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726154, 1467726154, 0, 0, 0, 0, 0, 0),
(24856, 24865, 'author', 'en', '[Live~HD] Sillamäe Kalev vs Trans Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726181, 1467726181, 0, 0, 0, 0, 0, 0),
(24857, 24866, 'author', 'en', '[Live~HD] SJK vs PK-35 Vantaa Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726201, 1467726201, 0, 0, 0, 0, 0, 0),
(24858, 24867, 'author', 'en', '[Live~HD] VPS vs Lahti Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726233, 1467726233, 0, 0, 0, 0, 0, 0),
(24859, 24868, 'author', 'en', '[Live~HD] Kiffen vs FC Espoo Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726249, 1467726249, 0, 0, 0, 0, 0, 0),
(24860, 24869, 'author', 'en', '[Live~HD] Akzhayik vs Astana Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726275, 1467726275, 0, 0, 0, 0, 0, 0),
(24861, 24870, 'author', 'en', '[Live~HD] Liepājas Metalurgs vs BFC Daugavpils Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726296, 1467726296, 0, 0, 0, 0, 0, 0),
(24862, 24871, 'author', 'en', '[Live~HD] Start vs Bodø / Glimt Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726319, 1467726319, 0, 0, 0, 0, 0, 0),
(24863, 24872, 'author', 'en', '[Live~HD] Babelsberg vs St. Pauli Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726342, 1467726342, 0, 0, 0, 0, 0, 0),
(24864, 24873, 'author', 'en', '[Live~HD] Basel vs Lausanne Sport Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726364, 1467726364, 0, 0, 0, 0, 0, 0),
(24865, 24874, 'author', 'en', '[Live~HD] Weiche Flensburg vs Hamburger SV Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726387, 1467726387, 0, 0, 0, 0, 0, 0),
(24866, 24875, 'author', 'en', '[Live~HD] Erkenschwick vs Borussia Dortmund Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726402, 1467726402, 0, 0, 0, 0, 0, 0),
(24867, 24876, 'author', 'en', '[Live~HD] Tranmere Rovers vs Liverpool Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1467726438, 1467726438, 0, 0, 0, 0, 0, 0),
(24868, 24877, 'author', 'en', 'heunceut2016', 1970, 1, 1467726969, 1467726969, 0, 0, 0, 0, 0, 0),
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(24871, 24880, 'author', 'en', 'hd beauty! Pretty Little Liars Season 7 Episode 3 Online Full S07E03', 1974, 1, 1467728039, 1467728039, 0, 0, 0, 0, 0, 0),
(24872, 24881, 'author', 'en', 'Putlocker Pretty Little Liars Season 7 Episode 3 (2016) Online S07E03', 1974, 1, 1467728485, 1467728485, 0, 0, 0, 0, 0, 0),
(24873, 24882, 'author', 'en', 'Watch!! Dominika Cibulkova vs Elena Vesnina Live Stream Tennis 2016', 1975, 1, 1467728798, 1467728798, 0, 0, 0, 0, 0, 0),
(24874, 24883, 'author', 'en', 'No Script Zolpidem', 1888, 1, 1467729856, 1467729856, 0, 0, 0, 0, 0, 0),
(24875, 24884, 'author', 'en', 'Limited! Wentworth Season 4 Episode 9 Online Full S04E09 ', 1974, 1, 1467730763, 1467730763, 0, 0, 0, 0, 0, 0),
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(25010, 25019, 'author', 'en', '[The Beauty and the Beast] 4x6 Season 4 Episode 6 Watch Full Episodes Online', 2020, 1, 1467901693, 1467905261, 0, 0, 0, 0, 0, 0),
(25011, 25020, 'author', 'en', '[Beast of Times, Worst of Times] The Beauty and the Beast 4-6 Season 4 Episode 6 Full Videos Online', 2020, 1, 1467901749, 1467901749, 0, 0, 0, 0, 0, 0),
(25012, 25021, 'author', 'en', '[Beast of Times, Worst of Times] The Beauty and the Beast 4-6 Season 4 Episode 6 Full Videos Online free', 2020, 1, 1467901843, 1467905338, 0, 0, 0, 0, 0, 0),
(25013, 25022, 'author', 'en', '||The Beauty and the Beast|| (@TVD) Season 4 Episode 6 Watch Full Episodes', 2020, 1, 1467902017, 1467905369, 0, 0, 0, 0, 0, 0),
(25014, 25023, 'author', 'en', '[FREE@] The Beauty and the Beast 4x6 Season 4 Episode 6 Stream Full Online', 2020, 1, 1467902184, 1467905435, 0, 0, 0, 0, 0, 0),
(25015, 25024, 'author', 'en', 'The Beauty and the Beast 4x6 [Season 4 Episode 6] Live Stream The CW Online', 2020, 1, 1467902404, 1467905498, 0, 0, 0, 0, 0, 0),
(25016, 25025, 'author', 'en', 'Maria', 2031, 1, 1467904839, 1467904839, 0, 0, 0, 0, 0, 0),
(25017, 25026, 'author', 'en', 'Buy Online Tenormin Moneygram', 1760, 1, 1467905451, 1467905451, 0, 0, 0, 0, 0, 0),
(25018, 25027, 'author', 'en', 'Maria Hill', 2031, 1, 1467905840, 1467905840, 0, 0, 0, 0, 0, 0),
(25019, 25028, 'author', 'en', '1', 2031, 1, 1467906086, 1467906086, 0, 0, 0, 0, 0, 0),
(25020, 25029, 'author', 'en', 'V zajetí démonc 2 Stream zdarma ceštinae', 2031, 1, 1467906194, 1467906194, 0, 0, 0, 0, 0, 0),
(25021, 25030, 'author', 'en', 'Správní chlapi Stream zdarma ceštinae', 2031, 1, 1467906430, 1467906430, 0, 0, 0, 0, 0, 0),
(25022, 25031, 'author', 'en', 'Želvy Ninja 2 Stream zdarma ceštinae', 2031, 1, 1467906537, 1467906537, 0, 0, 0, 0, 0, 0),
(25023, 25032, 'author', 'en', 'Alenka v cíši divc: Za zrcadlem Stream zdarma ceštinae', 2031, 1, 1467906623, 1467906623, 0, 0, 0, 0, 0, 0),
(25024, 25033, 'author', 'en', 'Angry birds ve filmu Stream zdarma ceštinae', 2031, 1, 1467906689, 1467906689, 0, 0, 0, 0, 0, 0),
(25025, 25034, 'author', 'en', 'X-Men: Apokalypsa Stream zdarma ceštinae', 2031, 1, 1467906751, 1467906751, 0, 0, 0, 0, 0, 0),
(25026, 25035, 'author', 'en', 'Kniha džungle Stream zdarma ceštinae', 2031, 1, 1467906819, 1467906819, 0, 0, 0, 0, 0, 0),
(25027, 25036, 'author', 'en', 'Captain America: Obcanská válka Stream zdarma ceštinae', 2031, 1, 1467906892, 1467906892, 0, 0, 0, 0, 0, 0),
(25028, 25037, 'author', 'en', 'Svátek matek Stream zdarma ceštinae', 2031, 1, 1467906966, 1467906966, 0, 0, 0, 0, 0, 0),
(25029, 25038, 'author', 'en', 'Návštcvníci 3: Revoluce Stream zdarma ceštinae', 2031, 1, 1467907039, 1467907039, 0, 0, 0, 0, 0, 0),
(25030, 25039, 'author', 'en', 'Sousedi 2 Stream zdarma ceštinae', 2031, 1, 1467907101, 1467907101, 0, 0, 0, 0, 0, 0),
(25031, 25040, 'author', 'en', 'Jak pcežít single Stream zdarma ceštinae', 2031, 1, 1467907161, 1467907161, 0, 0, 0, 0, 0, 0),
(25032, 25041, 'author', 'en', 'Zootropolis: Mcsto zvícat Stream zdarma ceštinae', 2031, 1, 1467907336, 1467907336, 0, 0, 0, 0, 0, 0),
(25033, 25042, 'author', 'en', 'Oslncni sluncem Stream zdarma ceštinae', 2031, 1, 1467907442, 1467907442, 0, 0, 0, 0, 0, 0),
(25034, 25043, 'author', 'en', 'Vzkaz v láhvi Stream zdarma ceštinae', 2031, 1, 1467907521, 1467907521, 0, 0, 0, 0, 0, 0),
(25035, 25044, 'author', 'en', 'Maggie má plán Stream zdarma ceštinae', 2031, 1, 1467907622, 1467907622, 0, 0, 0, 0, 0, 0),
(25036, 25045, 'author', 'en', 'Kolonie Stream zdarma ceštinae', 2031, 1, 1467907684, 1467907684, 0, 0, 0, 0, 0, 0),
(25037, 25046, 'author', 'en', 'Pat a Mat ve filmu Stream zdarma ceštinae', 2031, 1, 1467907775, 1467907775, 0, 0, 0, 0, 0, 0),
(25038, 25047, 'author', 'en', 'Kung Fu Panda 3 Stream zdarma ceštinae', 2031, 1, 1467907850, 1467907850, 0, 0, 0, 0, 0, 0),
(25039, 25048, 'author', 'en', 'Szemfényvesztok 2 folyam szabad magyar', 2031, 1, 1467907929, 1467907929, 0, 0, 0, 0, 0, 0),
(25040, 25049, 'author', 'en', 'Pénzes cápa folyam szabad magyar', 2031, 1, 1467908025, 1467908025, 0, 0, 0, 0, 0, 0),
(25041, 25050, 'author', 'en', 'Alice Tükörországban folyam szabad magyar', 2031, 1, 1467908107, 1467908107, 0, 0, 0, 0, 0, 0),
(25042, 25051, 'author', 'en', 'Rendes fickók folyam szabad magyar', 2031, 1, 1467908221, 1467908221, 0, 0, 0, 0, 0, 0),
(25043, 25052, 'author', 'en', 'Warcraft: A kezdetek folyam szabad magyar', 2031, 1, 1467908316, 1467908316, 0, 0, 0, 0, 0, 0),
(25044, 25053, 'author', 'en', 'Démonok között 2. folyam szabad magyar', 2031, 1, 1467908410, 1467908410, 0, 0, 0, 0, 0, 0),
(25045, 25054, 'author', 'en', 'X-Men - Apokalipszis folyam szabad magyar', 2031, 1, 1467908488, 1467908488, 0, 0, 0, 0, 0, 0),
(25046, 25055, 'author', 'en', 'Angry Birds - A film folyam szabad magyar', 2031, 1, 1467908588, 1467908588, 0, 0, 0, 0, 0, 0),
(25047, 25056, 'author', 'en', 'Amerika Kapitány: Polgárháború folyam szabad magyar', 2031, 1, 1467908674, 1467908674, 0, 0, 0, 0, 0, 0),
(25048, 25057, 'author', 'en', 'A dzsungel könyve folyam szabad magyar', 2031, 1, 1467908763, 1467908763, 0, 0, 0, 0, 0, 0),
(25049, 25058, 'author', 'en', 'Szenilla nyomában folyam szabad magyar', 2031, 1, 1467908839, 1467908839, 0, 0, 0, 0, 0, 0),
(25050, 25059, 'author', 'en', 'A legkúlabb nap folyam szabad magyar', 2031, 1, 1467908906, 1467908906, 0, 0, 0, 0, 0, 0),
(25051, 25060, 'author', 'en', 'Rossz szomszédság 2 folyam szabad magyar', 2031, 1, 1467908974, 1467908974, 0, 0, 0, 0, 0, 0),
(25052, 25061, 'author', 'en', 'Csodálatos Boccaccio folyam szabad magyar', 2031, 1, 1467909046, 1467909046, 0, 0, 0, 0, 0, 0),
(25053, 25062, 'author', 'en', 'Máris hiányzol folyam szabad magyar', 2031, 1, 1467909115, 1467909115, 0, 0, 0, 0, 0, 0),
(25054, 25063, 'author', 'en', 'Pelé folyam szabad magyar', 2031, 1, 1467909186, 1467909186, 0, 0, 0, 0, 0, 0),
(25055, 25064, 'author', 'en', 'Anyák napja folyam szabad magyar', 2031, 1, 1467909256, 1467909256, 0, 0, 0, 0, 0, 0),
(25056, 25065, 'author', 'en', 'Évszakok folyam szabad magyar', 2031, 1, 1467909332, 1467909332, 0, 0, 0, 0, 0, 0),
(25057, 25066, 'author', 'en', 'Zootropolis - Állati nagy balhé folyam szabad magyar', 2031, 1, 1467909406, 1467909406, 0, 0, 0, 0, 0, 0),
(25058, 25067, 'author', 'en', 'Központi hírszerzés folyam szabad magyar', 2031, 1, 1467909476, 1467909476, 0, 0, 0, 0, 0, 0),
(25059, 25068, 'author', 'en', 'A szerelem gyerekkel jön folyam szabad magyar', 2031, 1, 1467909552, 1467909552, 0, 0, 0, 0, 0, 0),
(25060, 25069, 'author', 'en', 'Haifai kiköt? folyam szabad magyar', 2031, 1, 1467909624, 1467909624, 0, 0, 0, 0, 0, 0),
(25061, 25070, 'author', 'en', 'Egy ?rült pillanat folyam szabad magyar', 2031, 1, 1467909700, 1467909700, 0, 0, 0, 0, 0, 0),
(25062, 25071, 'author', 'en', 'Ha Isten úgy akarja folyam szabad magyar', 2031, 1, 1467909770, 1467909770, 0, 0, 0, 0, 0, 0),
(25063, 25072, 'author', 'en', 'Kék bársony folyam szabad magyar', 2031, 1, 1467909841, 1467909841, 0, 0, 0, 0, 0, 0),
(25064, 25073, 'author', 'en', 'Egy szó, mint száz: szerelem! folyam szabad magyar', 2031, 1, 1467909927, 1467909927, 0, 0, 0, 0, 0, 0),
(25065, 25074, 'author', 'en', 'Elcserélt világ folyam szabad magyar', 2031, 1, 1467909996, 1467909996, 0, 0, 0, 0, 0, 0),
(25066, 25075, 'author', 'en', 'Buy Now Voltaren-Gel Tabs', 1888, 1, 1467910033, 1467910033, 0, 0, 0, 0, 0, 0),
(25067, 25076, 'author', 'en', 'A kommuna folyam szabad magyar', 2031, 1, 1467910064, 1467910064, 0, 0, 0, 0, 0, 0),
(25068, 25077, 'author', 'en', 'Cloverfield Lane 10 folyam szabad magyar', 2031, 1, 1467910134, 1467910134, 0, 0, 0, 0, 0, 0),
(25069, 25078, 'author', 'en', 'A F?nök folyam szabad magyar', 2031, 1, 1467910210, 1467910210, 0, 0, 0, 0, 0, 0),
(25070, 25079, 'author', 'en', 'Robinson Crusoe folyam szabad magyar', 2031, 1, 1467910284, 1467910284, 0, 0, 0, 0, 0, 0),
(25071, 25080, 'author', 'en', 'Kung Fu Panda 3 folyam szabad magyar', 2031, 1, 1467910353, 1467910353, 0, 0, 0, 0, 0, 0),
(25072, 25081, 'author', 'en', 'Spotlight - Egy nyomozás részletei folyam szabad magyar', 2031, 1, 1467910427, 1467910427, 0, 0, 0, 0, 0, 0),
(25073, 25082, 'author', 'en', 'Ifjúság folyam szabad magyar', 2031, 1, 1467910498, 1467910498, 0, 0, 0, 0, 0, 0),
(25074, 25083, 'author', 'en', 'Miel?tt megismertelek folyam szabad magyar', 2031, 1, 1467911110, 1467911110, 0, 0, 0, 0, 0, 0),
(25075, 25084, 'author', 'en', 'Izzie nyomában folyam szabad magyar', 2031, 1, 1467911155, 1467911155, 0, 0, 0, 0, 0, 0),
(25076, 25085, 'author', 'en', 'A lánykirály folyam szabad magyar', 2031, 1, 1467911198, 1467911198, 0, 0, 0, 0, 0, 0),
(25077, 25086, 'author', 'en', 'Ben Hur folyam szabad magyar', 2031, 1, 1467911242, 1467911242, 0, 0, 0, 0, 0, 0),
(25078, 25087, 'author', 'en', 'Ratchet és Clank - A galaxis védelmez?i folyam szabad magyar', 2031, 1, 1467911289, 1467911289, 0, 0, 0, 0, 0, 0),
(25079, 25088, 'author', 'en', 'Én és Kaminski folyam szabad magyar', 2031, 1, 1467911332, 1467911332, 0, 0, 0, 0, 0, 0),
(25080, 25089, 'author', 'en', 'Madárkák folyam szabad magyar', 2031, 1, 1467911382, 1467911382, 0, 0, 0, 0, 0, 0),
(25081, 25090, 'author', 'en', 'Bazi nagy görög lagzi 2. folyam szabad magyar', 2031, 1, 1467911434, 1467911434, 0, 0, 0, 0, 0, 0),
(25082, 25091, 'author', 'en', 'Vad Kunság - A Puszta rejtett élete folyam szabad magyar', 2031, 1, 1467911473, 1467911473, 0, 0, 0, 0, 0, 0),
(25083, 25092, 'author', 'en', 'Férfiak és csirkék folyam szabad magyar', 2031, 1, 1467911513, 1467911513, 0, 0, 0, 0, 0, 0),
(25084, 25093, 'author', 'en', 'A szoba folyam szabad magyar', 2031, 1, 1467911553, 1467911553, 0, 0, 0, 0, 0, 0),
(25085, 25094, 'author', 'en', 'Az én Szerelmem folyam szabad magyar', 2031, 1, 1467911594, 1467911594, 0, 0, 0, 0, 0, 0),
(25086, 25095, 'author', 'en', '45 év folyam szabad magyar', 2031, 1, 1467911635, 1467911635, 0, 0, 0, 0, 0, 0),
(25087, 25096, 'author', 'en', 'A lakás folyam szabad magyar', 2031, 1, 1467911674, 1467911674, 0, 0, 0, 0, 0, 0),
(25088, 25097, 'author', 'en', 'A halott menyasszony folyam szabad magyar', 2031, 1, 1467911715, 1467911715, 0, 0, 0, 0, 0, 0),
(25089, 25098, 'author', 'en', 'Magyar csapat - "...még 50 perc..." folyam szabad magyar', 2031, 1, 1467911756, 1467911756, 0, 0, 0, 0, 0, 0),
(25090, 25099, 'author', 'en', 'Sarkköri ment?expedíció folyam szabad magyar', 2031, 1, 1467911796, 1467911796, 0, 0, 0, 0, 0, 0),
(25091, 25100, 'author', 'en', 'A Vadász és a Jégkirályn? folyam szabad magyar', 2031, 1, 1467911836, 1467911836, 0, 0, 0, 0, 0, 0),
(25092, 25101, 'author', 'en', 'Jöttünk, láttunk, visszamennénk 3. folyam szabad magyar', 2031, 1, 1467911877, 1467911877, 0, 0, 0, 0, 0, 0),
(25093, 25102, 'author', 'en', 'Mama folyam szabad magyar', 2031, 1, 1467911915, 1467911915, 0, 0, 0, 0, 0, 0),
(25094, 25103, 'author', 'en', 'Batman Superman ellen - Az igazság hajnala folyam szabad magyar', 2031, 1, 1467911954, 1467911954, 0, 0, 0, 0, 0, 0),
(25095, 25104, 'author', 'en', 'A Beavatott-sorozat: A h?séges folyam szabad magyar', 2031, 1, 1467911995, 1467911995, 0, 0, 0, 0, 0, 0),
(25096, 25105, 'author', 'en', 'A Keresztapa folyam szabad magyar', 2031, 1, 1467912037, 1467912037, 0, 0, 0, 0, 0, 0),
(25097, 25106, 'author', 'en', 'Hétköznapi titkaink folyam szabad magyar', 2031, 1, 1467912075, 1467912075, 0, 0, 0, 0, 0, 0),
(25098, 25107, 'author', 'en', 'Csokoládé folyam szabad magyar', 2031, 1, 1467912115, 1467912115, 0, 0, 0, 0, 0, 0),
(25099, 25108, 'author', 'en', 'Mennyit ér egy ember? folyam szabad magyar', 2031, 1, 1467912154, 1467912154, 0, 0, 0, 0, 0, 0),
(25100, 25109, 'author', 'en', 'Egyiptom istenei folyam szabad magyar', 2031, 1, 1467912195, 1467912195, 0, 0, 0, 0, 0, 0),
(25101, 25110, 'author', 'en', 'A Zarándok - Paulo Coelho legjobb története folyam szabad magyar', 2031, 1, 1467912236, 1467912236, 0, 0, 0, 0, 0, 0),
(25102, 25111, 'author', 'en', 'Deadpool folyam szabad magyar', 2031, 1, 1467912277, 1467912277, 0, 0, 0, 0, 0, 0),
(25103, 25112, 'author', 'en', 'A boldogság titka folyam szabad magyar', 2031, 1, 1467912318, 1467912318, 0, 0, 0, 0, 0, 0),
(25104, 25113, 'author', 'en', 'Alvin és a mókusok - A mókás menet folyam szabad magyar', 2031, 1, 1467912361, 1467912361, 0, 0, 0, 0, 0, 0),
(25105, 25114, 'author', 'en', 'Lépcs?házi történetek folyam szabad magyar', 2031, 1, 1467912401, 1467912401, 0, 0, 0, 0, 0, 0),
(25106, 25115, 'author', 'en', 'Truman folyam szabad magyar', 2031, 1, 1467912440, 1467912440, 0, 0, 0, 0, 0, 0),
(25107, 25116, 'author', 'en', 'Mustang folyam szabad magyar', 2031, 1, 1467912480, 1467912480, 0, 0, 0, 0, 0, 0),
(25108, 25117, 'author', 'en', 'Az él?halottak éjszakája folyam szabad magyar', 2031, 1, 1467912521, 1467912521, 0, 0, 0, 0, 0, 0),
(25109, 25118, 'author', 'en', 'A kis herceg folyam szabad magyar', 2031, 1, 1467912561, 1467912561, 0, 0, 0, 0, 0, 0),
(25110, 25119, 'author', 'en', 'Liza, a rókatündér folyam szabad magyar', 2031, 1, 1467912602, 1467912602, 0, 0, 0, 0, 0, 0),
(25111, 25120, 'author', 'en', 'Legújabb testamentum folyam szabad magyar', 2031, 1, 1467912642, 1467912642, 0, 0, 0, 0, 0, 0),
(25112, 25121, 'author', 'en', 'Csodapirula folyam szabad magyar', 2031, 1, 1467912681, 1467912681, 0, 0, 0, 0, 0, 0),
(25113, 25122, 'author', 'en', 'Münó, a holdbéli manó folyam szabad magyar', 2031, 1, 1467912726, 1467912726, 0, 0, 0, 0, 0, 0),
(25114, 25123, 'author', 'en', 'Szerelem Máltán folyam szabad magyar', 2031, 1, 1467912765, 1467912765, 0, 0, 0, 0, 0, 0),
(25115, 25124, 'author', 'en', 'Éjfélkor folyam szabad magyar', 2031, 1, 1467912803, 1467912803, 0, 0, 0, 0, 0, 0),
(25116, 25125, 'author', 'en', 'Szavak nélkül folyam szabad magyar', 2031, 1, 1467912842, 1467912842, 0, 0, 0, 0, 0, 0),
(25117, 25126, 'author', 'en', 'Leviatán folyam szabad magyar', 2031, 1, 1467912880, 1467912880, 0, 0, 0, 0, 0, 0),
(25118, 25127, 'author', 'en', 'Fehér tenyér folyam szabad magyar', 2031, 1, 1467912917, 1467912917, 0, 0, 0, 0, 0, 0),
(25119, 25128, 'author', 'en', 'Külvárosi legenda folyam szabad magyar', 2031, 1, 1467912955, 1467912955, 0, 0, 0, 0, 0, 0),
(25120, 25129, 'author', 'en', 'Kochaj strumien darmowe Polskie', 2031, 1, 1467912995, 1467912995, 0, 0, 0, 0, 0, 0),
(25121, 25130, 'author', 'en', 'Zanim si? pojawi?e? strumie? darmowe Polskie', 2031, 1, 1467913035, 1467913035, 0, 0, 0, 0, 0, 0),
(25122, 25131, 'author', 'en', 'Angry Birds Film strumie? darmowe Polskie', 2031, 1, 1467913072, 1467913072, 0, 0, 0, 0, 0, 0),
(25123, 25132, 'author', 'en', 'Buying Ortho Tri-Cyclen No Prescription', 1760, 1, 1467913081, 1467913081, 0, 0, 0, 0, 0, 0),
(25124, 25133, 'author', 'en', 'Nice Guys. Równi go?cie strumie? darmowe Polskie', 2031, 1, 1467913111, 1467913111, 0, 0, 0, 0, 0, 0),
(25125, 25134, 'author', 'en', 'Warcraft: Pocz?tek strumie? darmowe Polskie', 2031, 1, 1467913149, 1467913149, 0, 0, 0, 0, 0, 0),
(25126, 25135, 'author', 'en', 'The Conjuring 2 strumie? darmowe Polskie', 2031, 1, 1467913190, 1467913190, 0, 0, 0, 0, 0, 0),
(25127, 25136, 'author', 'en', 'Wojownicze ?ó?wie ninja: Wyj?cie z cienia strumie? darmowe Polskie', 2031, 1, 1467913232, 1467913232, 0, 0, 0, 0, 0, 0),
(25128, 25137, 'author', 'en', 'Dzie? Bastylii strumie? darmowe Polskie', 2031, 1, 1467913274, 1467913274, 0, 0, 0, 0, 0, 0),
(25129, 25138, 'author', 'en', 'Zak?adnik z Wall Street strumie? darmowe Polskie', 2031, 1, 1467913312, 1467913312, 0, 0, 0, 0, 0, 0),
(25130, 25139, 'author', 'en', 'Idol z ulicy strumie? darmowe Polskie', 2031, 1, 1467913350, 1467913350, 0, 0, 0, 0, 0, 0),
(25131, 25140, 'author', 'en', 'Jak uratowa? mam? strumie? darmowe Polskie', 2031, 1, 1467913393, 1467913393, 0, 0, 0, 0, 0, 0),
(25132, 25141, 'author', 'en', 'X-Men: Apocalypse strumie? darmowe Polskie', 2031, 1, 1467913434, 1467913434, 0, 0, 0, 0, 0, 0),
(25133, 25142, 'author', 'en', 'Kapitan Ameryka: Wojna bohaterów strumie? darmowe Polskie', 2031, 1, 1467913475, 1467913475, 0, 0, 0, 0, 0, 0),
(25134, 25143, 'author', 'en', 'Ksi?ga d?ungli strumie? darmowe Polskie', 2031, 1, 1467913521, 1467913521, 0, 0, 0, 0, 0, 0),
(25135, 25144, 'author', 'en', 'Alicja po drugiej stronie lustra strumie? darmowe Polskie', 2031, 1, 1467913560, 1467913560, 0, 0, 0, 0, 0, 0),
(25136, 25145, 'author', 'en', 'Dzie? Matki strumie? darmowe Polskie', 2031, 1, 1467913598, 1467913598, 0, 0, 0, 0, 0, 0),
(25137, 25146, 'author', 'en', 'Zanim si? obudz? strumie? darmowe Polskie', 2031, 1, 1467913637, 1467913637, 0, 0, 0, 0, 0, 0),
(25138, 25147, 'author', 'en', 'S?siedzi 215 strumie? darmowe Polskie', 2031, 1, 1467913675, 1467913675, 0, 0, 0, 0, 0, 0),
(25139, 25148, 'author', 'en', 'Kung Fu Panda 3 strumie? darmowe Polskie', 2031, 1, 1467913714, 1467913714, 0, 0, 0, 0, 0, 0),
(25140, 25149, 'author', 'en', 'Mój przyjaciel orze? strumie? darmowe Polskie', 2031, 1, 1467913756, 1467913756, 0, 0, 0, 0, 0, 0),
(25141, 25150, 'author', 'en', 'Zwierzogród strumie? darmowe Polskie', 2031, 1, 1467913795, 1467913795, 0, 0, 0, 0, 0, 0),
(25142, 25151, 'author', 'en', 'Dzikie z natury strumie? darmowe Polskie', 2031, 1, 1467913834, 1467913834, 0, 0, 0, 0, 0, 0),
(25143, 25152, 'author', 'en', 'Ojciec strumie? darmowe Polskie', 2031, 1, 1467913873, 1467913873, 0, 0, 0, 0, 0, 0),
(25144, 25153, 'author', 'en', 'Szukaj?c Kelly strumie? darmowe Polskie', 2031, 1, 1467913912, 1467913912, 0, 0, 0, 0, 0, 0),
(25145, 25154, 'author', 'en', 'Wojna strumie? darmowe Polskie', 2031, 1, 1467913951, 1467913951, 0, 0, 0, 0, 0, 0),
(25146, 25155, 'author', 'en', 'Bóg w Krakowie strumie? darmowe Polskie', 2031, 1, 1467913991, 1467913991, 0, 0, 0, 0, 0, 0),
(25147, 25156, 'author', 'en', 'Baby Bump strumie? darmowe Polskie', 2031, 1, 1467914030, 1467914030, 0, 0, 0, 0, 0, 0),
(25148, 25157, 'author', 'en', 'Summer Camp strumie? darmowe Polskie', 2031, 1, 1467914069, 1467914069, 0, 0, 0, 0, 0, 0),
(25149, 25158, 'author', 'en', 'Opiekun strumie? darmowe Polskie', 2031, 1, 1467914109, 1467914109, 0, 0, 0, 0, 0, 0),
(25150, 25159, 'author', 'en', 'Dzielny kogut Maniek strumie? darmowe Polskie', 2031, 1, 1467914148, 1467914148, 0, 0, 0, 0, 0, 0),
(25151, 25160, 'author', 'en', 'Specjalny program strumie? darmowe Polskie', 2031, 1, 1467914187, 1467914187, 0, 0, 0, 0, 0, 0),
(25152, 25161, 'author', 'en', 'Pod tafl? oceanu strumie? darmowe Polskie', 2031, 1, 1467914228, 1467914228, 0, 0, 0, 0, 0, 0),
(25153, 25162, 'author', 'en', 'Blokada strumie? darmowe Polskie', 2031, 1, 1467914267, 1467914267, 0, 0, 0, 0, 0, 0),
(25154, 25163, 'author', 'en', 'Królowa strumie? darmowe Polskie', 2031, 1, 1467914310, 1467914310, 0, 0, 0, 0, 0, 0),
(25155, 25164, 'author', 'en', 'Cyrk w cyrku strumie? darmowe Polskie', 2031, 1, 1467914352, 1467914352, 0, 0, 0, 0, 0, 0),
(25156, 25165, 'author', 'en', 'Ostatni kurs strumie? darmowe Polskie', 2031, 1, 1467914393, 1467914393, 0, 0, 0, 0, 0, 0),
(25157, 25166, 'author', 'en', 'Skarb strumie? darmowe Polskie', 2031, 1, 1467914431, 1467914431, 0, 0, 0, 0, 0, 0),
(25158, 25167, 'author', 'en', 'How to Plan an Orgy in a Small Town strumie? darmowe Polskie', 2031, 1, 1467914471, 1467914471, 0, 0, 0, 0, 0, 0),
(25159, 25168, 'author', 'en', 'Lato Sangaile strumie? darmowe Polskie', 2031, 1, 1467914510, 1467914510, 0, 0, 0, 0, 0, 0),
(25160, 25169, 'author', 'en', 'Nienasyceni strumie? darmowe Polskie', 2031, 1, 1467914549, 1467914549, 0, 0, 0, 0, 0, 0),
(25161, 25170, 'author', 'en', 'By?y sobie cz?owieki strumie? darmowe Polskie', 2031, 1, 1467914590, 1467914590, 0, 0, 0, 0, 0, 0),
(25162, 25171, 'author', 'en', 'M?ody Mesjasz strumie? darmowe Polskie', 2031, 1, 1467914629, 1467914629, 0, 0, 0, 0, 0, 0),
(25163, 25172, 'author', 'en', 'Music Program strumie? darmowe Polskie', 2031, 1, 1467914668, 1467914668, 0, 0, 0, 0, 0, 0),
(25164, 25173, 'author', 'en', 'Pitbull. Nowe porzadki strumie? darmowe Polskie', 2031, 1, 1467914708, 1467914708, 0, 0, 0, 0, 0, 0),
(25165, 25174, 'author', 'en', 'Papusza strumie? darmowe Polskie', 2031, 1, 1467914746, 1467914746, 0, 0, 0, 0, 0, 0),
(25166, 25175, 'author', 'en', 'Afryka?ska przygoda 3D - safari nad Okavango darmowe Polskie', 2031, 1, 1467914786, 1467914786, 0, 0, 0, 0, 0, 0),
(25167, 25176, 'author', 'en', 'Królestwo zwierz?t strumie? darmowe Polskie', 2031, 1, 1467914826, 1467914826, 0, 0, 0, 0, 0, 0),
(25168, 25177, 'author', 'en', 'Playtime strumie? darmowe Polskie', 2031, 1, 1467914864, 1467914864, 0, 0, 0, 0, 0, 0),
(25169, 25178, 'author', 'en', 'Miles Ahead Stream Gratis lëtzebuergesch', 2031, 1, 1467914906, 1467914906, 0, 0, 0, 0, 0, 0),
(25170, 25179, 'author', 'en', 'Ninja Turtles Out of the Shadows (VO) Stream Gratis lëtzebuergesch', 2031, 1, 1467914944, 1467914944, 0, 0, 0, 0, 0, 0),
(25171, 25180, 'author', 'en', 'Ninja Turtles 2 (VF) Stream Gratis lëtzebuergesch', 2031, 1, 1467914985, 1467914985, 0, 0, 0, 0, 0, 0),
(25172, 25181, 'author', 'en', 'Camping 3 Stream Gratis lëtzebuergesch', 2031, 1, 1467915024, 1467915024, 0, 0, 0, 0, 0, 0),
(25173, 25182, 'author', 'en', 'ROH: Werther 2016 Stream Gratis lëtzebuergesch', 2031, 1, 1467915063, 1467915063, 0, 0, 0, 0, 0, 0),
(25174, 25183, 'author', 'en', 'Finding Dory Stream Gratis lëtzebuergesch', 2031, 1, 1467915101, 1467915101, 0, 0, 0, 0, 0, 0),
(25175, 25184, 'author', 'en', 'Bastille Day Stream Gratis lëtzebuergesch', 2031, 1, 1467915141, 1467915141, 0, 0, 0, 0, 0, 0),
(25176, 25185, 'author', 'en', 'A man called Ove Stream Gratis lëtzebuergesch', 2031, 1, 1467915180, 1467915180, 0, 0, 0, 0, 0, 0),
(25177, 25186, 'author', 'en', 'Tout de suite, maintenant Stream Gratis lëtzebuergesch', 2031, 1, 1467915219, 1467915219, 0, 0, 0, 0, 0, 0),
(25178, 25187, 'author', 'en', 'L''Outsider Stream Gratis lëtzebuergesch', 2031, 1, 1467915258, 1467915258, 0, 0, 0, 0, 0, 0),
(25179, 25188, 'author', 'en', 'Remember Stream Gratis lëtzebuergesch', 2031, 1, 1467915298, 1467915298, 0, 0, 0, 0, 0, 0),
(25180, 25189, 'author', 'en', 'Me Before You Stream Gratis lëtzebuergesch', 2031, 1, 1467915337, 1467915337, 0, 0, 0, 0, 0, 0),
(25181, 25190, 'author', 'en', 'Our Kind of Traitor Stream Gratis lëtzebuergesch', 2031, 1, 1467915377, 1467915377, 0, 0, 0, 0, 0, 0),
(25182, 25191, 'author', 'en', 'L''ideal Stream Gratis lëtzebuergesch', 2031, 1, 1467915417, 1467915417, 0, 0, 0, 0, 0, 0),
(25183, 25192, 'author', 'en', 'La Pazza Gioia (Folles de joie) Stream Gratis lëtzebuergesch', 2031, 1, 1467915456, 1467915456, 0, 0, 0, 0, 0, 0),
(25184, 25193, 'author', 'en', 'Ma Loute Stream Gratis lëtzebuergesch', 2031, 1, 1467915495, 1467915495, 0, 0, 0, 0, 0, 0),
(25185, 25194, 'author', 'en', 'The Conjuring 2 Stream Gratis lëtzebuergesch', 2031, 1, 1467915535, 1467915535, 0, 0, 0, 0, 0, 0),
(25186, 25195, 'author', 'en', 'The Neon Demon Stream Gratis lëtzebuergesch', 2031, 1, 1467915573, 1467915573, 0, 0, 0, 0, 0, 0),
(25187, 25196, 'author', 'en', 'Warcraft: The Beginning (VA) Stream Gratis lëtzebuergesch', 2031, 1, 1467915613, 1467915613, 0, 0, 0, 0, 0, 0),
(25188, 25197, 'author', 'en', 'Warcraft (VO) Stream Gratis lëtzebuergesch', 2031, 1, 1467915652, 1467915652, 0, 0, 0, 0, 0, 0),
(25189, 25198, 'author', 'en', 'Money Monster Stream Gratis lëtzebuergesch', 2031, 1, 1467915692, 1467915692, 0, 0, 0, 0, 0, 0),
(25190, 25199, 'author', 'en', 'Ils sont partout Stream Gratis lëtzebuergesch', 2031, 1, 1467915731, 1467915731, 0, 0, 0, 0, 0, 0),
(25191, 25200, 'author', 'en', 'Retour chez ma mere Stream Gratis lëtzebuergesch', 2031, 1, 1467915770, 1467915770, 0, 0, 0, 0, 0, 0),
(25192, 25201, 'author', 'en', 'Alice im Wunderland 2: Hinter den Spiegeln Stream Gratis lëtzebuergesch', 2031, 1, 1467915809, 1467915809, 0, 0, 0, 0, 0, 0),
(25193, 25202, 'author', 'en', 'Alice Through the Looking Glass Stream Gratis lëtzebuergesch', 2031, 1, 1467915849, 1467915849, 0, 0, 0, 0, 0, 0),
(25194, 25203, 'author', 'en', 'Mother''s Day Stream Gratis lëtzebuergesch', 2031, 1, 1467915888, 1467915888, 0, 0, 0, 0, 0, 0),
(25195, 25204, 'author', 'en', 'Elle Stream Gratis lëtzebuergesch', 2031, 1, 1467915927, 1467915927, 0, 0, 0, 0, 0, 0),
(25196, 25205, 'author', 'en', 'X-Men: Apocalypse (VO) Stream Gratis lëtzebuergesch', 2031, 1, 1467915967, 1467915967, 0, 0, 0, 0, 0, 0),
(25197, 25206, 'author', 'en', 'Julieta Stream Gratis lëtzebuergesch', 2031, 1, 1467916006, 1467916006, 0, 0, 0, 0, 0, 0),
(25198, 25207, 'author', 'en', 'The Nice Guys Stream Gratis lëtzebuergesch', 2031, 1, 1467916047, 1467916047, 0, 0, 0, 0, 0, 0),
(25199, 25208, 'author', 'en', 'The Angry Birds Movie Stream Gratis lëtzebuergesch', 2031, 1, 1467916086, 1467916086, 0, 0, 0, 0, 0, 0),
(25200, 25209, 'author', 'en', 'Cafe Society Stream Gratis lëtzebuergesch', 2031, 1, 1467916126, 1467916126, 0, 0, 0, 0, 0, 0),
(25201, 25210, 'author', 'en', 'Les malheurs de Sophie (Sophie''s Misfortunes) Gratis lëtzebuergesch', 2031, 1, 1467916166, 1467916166, 0, 0, 0, 0, 0, 0),
(25202, 25211, 'author', 'en', 'Rico, Oskar und der Diebstahlstein Stream Gratis lëtzebuergesch', 2031, 1, 1467916205, 1467916205, 0, 0, 0, 0, 0, 0),
(25203, 25212, 'author', 'en', 'Captain America: Civil War (VO) Stream Gratis lëtzebuergesch', 2031, 1, 1467916244, 1467916244, 0, 0, 0, 0, 0, 0),
(25204, 25213, 'author', 'en', 'Trumbo Stream Gratis lëtzebuergesch', 2031, 1, 1467916283, 1467916283, 0, 0, 0, 0, 0, 0),
(25205, 25214, 'author', 'en', 'Voices from Chernobyl (La Supplication) Stream Gratis lëtzebuergesch', 2031, 1, 1467916323, 1467916323, 0, 0, 0, 0, 0, 0),
(25206, 25215, 'author', 'en', 'Der Staat gegen Fritz Bauer Stream Gratis lëtzebuergesch', 2031, 1, 1467916362, 1467916362, 0, 0, 0, 0, 0, 0),
(25207, 25216, 'author', 'en', 'Foreign Affairs Stream Gratis lëtzebuergesch', 2031, 1, 1467916401, 1467916401, 0, 0, 0, 0, 0, 0),
(25208, 25217, 'author', 'en', 'The Jungle Book (VO) Stream Gratis lëtzebuergesch', 2031, 1, 1467916440, 1467916440, 0, 0, 0, 0, 0, 0),
(25209, 25218, 'author', 'en', 'Kung Fu Panda 3 (VA) Stream Gratis lëtzebuergesch', 2031, 1, 1467916480, 1467916480, 0, 0, 0, 0, 0, 0),
(25210, 25219, 'author', 'en', 'Zoomania Stream Gratis lëtzebuergesch', 2031, 1, 1467916519, 1467916519, 0, 0, 0, 0, 0, 0),
(25211, 25220, 'author', 'en', 'Nosferatu Stream Gratis lëtzebuergesch', 2031, 1, 1467916559, 1467916559, 0, 0, 0, 0, 0, 0),
(25212, 25221, 'author', 'en', 'Le Monde de Dory Stream Gratis lëtzebuergesch', 2031, 1, 1467916597, 1467916597, 0, 0, 0, 0, 0, 0),
(25213, 25222, 'author', 'en', '((LIVE))) germany vs france ver en vivo', 2011, 1, 1467916620, 1467916620, 0, 0, 0, 0, 0, 0),
(25214, 25223, 'author', 'en', 'Et helt halvt år (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916676, 1467916676, 0, 0, 0, 0, 0, 0),
(25215, 25224, 'author', 'en', 'The Nice Guys (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916716, 1467916716, 0, 0, 0, 0, 0, 0),
(25216, 25225, 'author', 'en', 'The Conjuring 2 (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916755, 1467916755, 0, 0, 0, 0, 0, 0),
(25217, 25226, 'author', 'en', 'The Wild Life (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916794, 1467916794, 0, 0, 0, 0, 0, 0),
(25218, 25227, 'author', 'en', 'Teenage Mutant Ninja Turtles Out of the Shadows film på nett gratis', 2031, 1, 1467916833, 1467916833, 0, 0, 0, 0, 0, 0),
(25219, 25228, 'author', 'en', 'Money Monster (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916873, 1467916873, 0, 0, 0, 0, 0, 0),
(25220, 25229, 'author', 'en', 'Warcraft (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467916912, 1467916912, 0, 0, 0, 0, 0, 0),
(25221, 25230, 'author', 'en', 'Alice Through the Looking Glass på nett gratis med norsk undertekster', 2031, 1, 1467916951, 1467916951, 0, 0, 0, 0, 0, 0),
(25222, 25231, 'author', 'en', 'Angry Birds Filmen film på nett gratis med norsk undertekster', 2031, 1, 1467916990, 1467916990, 0, 0, 0, 0, 0, 0),
(25223, 25232, 'author', 'en', 'Den lille prinsen film på nett gratis med norsk undertekster', 2031, 1, 1467917029, 1467917029, 0, 0, 0, 0, 0, 0),
(25224, 25233, 'author', 'en', 'A Perfect Day film på nett gratis med norsk undertekster', 2031, 1, 1467917068, 1467917068, 0, 0, 0, 0, 0, 0),
(25225, 25234, 'author', 'en', 'The Neon Demon film på nett gratis med norsk undertekster', 2031, 1, 1467917107, 1467917107, 0, 0, 0, 0, 0, 0),
(25226, 25235, 'author', 'en', 'Alt det vakre film på nett gratis med norsk undertekster', 2031, 1, 1467917146, 1467917146, 0, 0, 0, 0, 0, 0),
(25227, 25236, 'author', 'en', 'X-Men: Apocalypse film på nett gratis med norsk undertekster', 2031, 1, 1467917186, 1467917186, 0, 0, 0, 0, 0, 0),
(25228, 25237, 'author', 'en', 'Jungelboken film på nett gratis med norsk undertekster', 2031, 1, 1467917225, 1467917225, 0, 0, 0, 0, 0, 0),
(25229, 25238, 'author', 'en', 'Neighbors 2 Sorority Rising film på nett gratis med norsk undertekster', 2031, 1, 1467917265, 1467917265, 0, 0, 0, 0, 0, 0),
(25230, 25239, 'author', 'en', 'Marnie - min hemmelige venninne på nett gratis med norsk', 2031, 1, 1467917304, 1467917304, 0, 0, 0, 0, 0, 0),
(25231, 25240, 'author', 'en', '(((LIVE:)) France vs. Germany,LIVE Online Euro 2016 semifinals', 2011, 1, 1467917306, 1467917306, 0, 0, 0, 0, 0, 0),
(25232, 25241, 'author', 'en', 'Captain America: Civil War på nett gratis med norsk undertekster', 2031, 1, 1467917345, 1467917345, 0, 0, 0, 0, 0, 0),
(25233, 25242, 'author', 'en', 'Gråtass - gøy på landet! film på nett gratis med norsk undertekster', 2031, 1, 1467917383, 1467917383, 0, 0, 0, 0, 0, 0),
(25234, 25243, 'author', 'en', 'Grand Hotel (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917422, 1467917422, 0, 0, 0, 0, 0, 0),
(25235, 25244, 'author', 'en', 'germany france en direct streaming', 2011, 1, 1467917440, 1467917440, 0, 0, 0, 0, 0, 0),
(25236, 25245, 'author', 'en', 'Human (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917462, 1467917462, 0, 0, 0, 0, 0, 0),
(25237, 25246, 'author', 'en', 'Mother''s Day (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917501, 1467917501, 0, 0, 0, 0, 0, 0),
(25238, 25247, 'author', 'en', 'A Conspiracy of Faith (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917542, 1467917542, 0, 0, 0, 0, 0, 0),
(25239, 25248, 'author', 'en', 'Kung Fu Panda 3 (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917581, 1467917581, 0, 0, 0, 0, 0, 0),
(25240, 25249, 'author', 'en', '[LiveTV]#Germany vs France Live S-tream Semifinal EURO 2016', 1895, 1, 1467917591, 1467917591, 0, 0, 0, 0, 0, 0),
(25241, 25250, 'author', 'en', 'Now You See Me 2 (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917623, 1467917623, 0, 0, 0, 0, 0, 0),
(25242, 25251, 'author', 'en', 'Zelig (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917663, 1467917663, 0, 0, 0, 0, 0, 0),
(25243, 25252, 'author', 'en', 'Our Kind of Traitor (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917702, 1467917702, 0, 0, 0, 0, 0, 0),
(25244, 25253, 'author', 'en', 'Pyromanen (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917742, 1467917742, 0, 0, 0, 0, 0, 0),
(25245, 25254, 'author', 'en', 'Historien om den sjenerte giganten på nett gratis med norsk undertekster', 2031, 1, 1467917782, 1467917782, 0, 0, 0, 0, 0, 0),
(25246, 25255, 'author', 'en', 'Zootropolis (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917822, 1467917822, 0, 0, 0, 0, 0, 0),
(25247, 25256, 'author', 'en', '45 år (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917860, 1467917860, 0, 0, 0, 0, 0, 0),
(25248, 25257, 'author', 'en', 'Passasjeren (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917903, 1467917903, 0, 0, 0, 0, 0, 0),
(25249, 25258, 'author', 'en', 'En fisk ved navn Wanda (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917942, 1467917942, 0, 0, 0, 0, 0, 0),
(25250, 25259, 'author', 'en', 'France - Germany en direct streaming', 2011, 1, 1467917956, 1467917956, 0, 0, 0, 0, 0, 0),
(25251, 25260, 'author', 'en', 'Laura (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467917981, 1467917981, 0, 0, 0, 0, 0, 0),
(25252, 25261, 'author', 'en', 'La Camioneta film på nett gratis med norsk undertekster', 2031, 1, 1467918020, 1467918020, 0, 0, 0, 0, 0, 0),
(25253, 25262, 'author', 'en', 'Havanastation (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918061, 1467918061, 0, 0, 0, 0, 0, 0),
(25254, 25263, 'author', 'en', 'Buy Strattera Cod N', 1888, 1, 1467918070, 1467918070, 0, 0, 0, 0, 0, 0),
(25255, 25264, 'author', 'en', 'Chinese Take-Away (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918101, 1467918101, 0, 0, 0, 0, 0, 0),
(25256, 25265, 'author', 'en', 'Iggy Pop Live in Basel (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918141, 1467918141, 0, 0, 0, 0, 0, 0),
(25257, 25266, 'author', 'en', 'Love & Friendship (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918180, 1467918180, 0, 0, 0, 0, 0, 0),
(25258, 25267, 'author', 'en', 'AAIC: St. Peter''s and The Papal Basilicas of Rome på nett gratis med norsk', 2031, 1, 1467918219, 1467918219, 0, 0, 0, 0, 0, 0),
(25259, 25268, 'author', 'en', 'Internasjonalt Kortfilmprogram film på nett gratis med norsk undertekster', 2031, 1, 1467918259, 1467918259, 0, 0, 0, 0, 0, 0),
(25260, 25269, 'author', 'en', 'mecz++>>Niemcy - Francja transmisja na żywo ON-line 07.07.2016 ', 1991, 1, 1467918281, 1467918281, 0, 0, 0, 0, 0, 0),
(25261, 25270, 'author', 'en', 'Mitt hjertes tapte slag (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918299, 1467918299, 0, 0, 0, 0, 0, 0),
(25262, 25271, 'author', 'en', 'Tootsie (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918338, 1467918338, 0, 0, 0, 0, 0, 0),
(25263, 25272, 'author', 'en', 'En meget diskret helt (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918378, 1467918378, 0, 0, 0, 0, 0, 0),
(25264, 25273, 'author', 'en', 'Sunset Boulevard (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918418, 1467918418, 0, 0, 0, 0, 0, 0),
(25265, 25274, 'author', 'en', 'The Boss (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918459, 1467918459, 0, 0, 0, 0, 0, 0),
(25266, 25275, 'author', 'en', 'Huset (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918497, 1467918497, 0, 0, 0, 0, 0, 0),
(25267, 25276, 'author', 'en', 'The Idol (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918538, 1467918538, 0, 0, 0, 0, 0, 0),
(25268, 25277, 'author', 'en', 'Kampen om fjordane (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918577, 1467918577, 0, 0, 0, 0, 0, 0),
(25269, 25278, 'author', 'en', 'Welcome to Norway (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918617, 1467918617, 0, 0, 0, 0, 0, 0),
(25270, 25279, 'author', 'en', 'germany france en direct', 2011, 1, 1467918651, 1467918651, 0, 0, 0, 0, 0, 0),
(25271, 25280, 'author', 'en', 'Brooklyn (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918657, 1467918657, 0, 0, 0, 0, 0, 0),
(25272, 25281, 'author', 'en', 'Alvin og Gjengen: I Farta (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918696, 1467918696, 0, 0, 0, 0, 0, 0),
(25273, 25282, 'author', 'en', 'Birkebeinerne (2016) film på nett gratis med norsk undertekster', 2031, 1, 1467918736, 1467918736, 0, 0, 0, 0, 0, 0),
(25274, 25283, 'author', 'en', 'Arabian Nights - Volume 3 film på nett gratis med norsk undertekster', 2031, 1, 1467918775, 1467918775, 0, 0, 0, 0, 0, 0),
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(25526, 25536, 'author', 'en', '[Live~HD] Santa Rosa De Pucallpa vs Unión Tarapoto Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249393, 1468249393, 0, 0, 0, 0, 0, 0),
(25527, 25537, 'author', 'en', '[Live~HD] Volgar-Gazprom vs Fakel Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249651, 1468249651, 0, 0, 0, 0, 0, 0),
(25528, 25538, 'author', 'en', '[Live~HD] Spartak Moskva II vs Sibir Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249668, 1468249668, 0, 0, 0, 0, 0, 0),
(25529, 25539, 'author', 'en', '[Live~HD] Tambov vs Ska Khabarovsk Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249698, 1468249698, 0, 0, 0, 0, 0, 0),
(25530, 25540, 'author', 'en', '[Live~HD] Spartak Nal''chik vs Kuban'' Krasnodar Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249750, 1468249750, 0, 0, 0, 0, 0, 0),
(25531, 25541, 'author', 'en', '[Live~HD] Tosno vs Khimki Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249773, 1468249773, 0, 0, 0, 0, 0, 0),
(25532, 25542, 'author', 'en', '[Live~HD] Zenit II vs Sokol Saratov Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249790, 1468249790, 0, 0, 0, 0, 0, 0),
(25533, 25543, 'author', 'en', '[Live~HD] Dinamo Moskva vs Tyumen Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249812, 1468249812, 0, 0, 0, 0, 0, 0),
(25534, 25544, 'author', 'en', '[Live~HD] Baltika vs Shinnik Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249837, 1468249837, 0, 0, 0, 0, 0, 0),
(25535, 25545, 'author', 'en', '[Live~HD] Eskilstuna UTD Women vs Umea Ik Women Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249882, 1468249882, 0, 0, 0, 0, 0, 0),
(25536, 25546, 'author', 'en', '[Live~HD] Malmö FF vs Örebro Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249906, 1468249906, 0, 0, 0, 0, 0, 0),
(25537, 25547, 'author', 'en', '[Live~HD] Oddevold vs Norrby Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249923, 1468249923, 0, 0, 0, 0, 0, 0),
(25538, 25548, 'author', 'en', '[Live~HD] Bethlehem Steel vs Pittsburgh Riverhounds Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249939, 1468249939, 0, 0, 0, 0, 0, 0),
(25539, 25549, 'author', 'en', '[Live~HD] City Islanders vs Toronto II Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249968, 1468249968, 0, 0, 0, 0, 0, 0),
(25540, 25550, 'author', 'en', '[Live~HD] Rochester Rhinos vs Orlando City II Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468249985, 1468249985, 0, 0, 0, 0, 0, 0),
(25541, 25551, 'author', 'en', '[Live~HD] Orange County Blues vs Sacramento Republic Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250001, 1468250001, 0, 0, 0, 0, 0, 0),
(25542, 25552, 'author', 'en', '[Live~HD] Seattle Sounders II vs Colorado Springs Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250025, 1468250025, 0, 0, 0, 0, 0, 0),
(25543, 25553, 'author', 'en', '[Live~HD] Rio Grande Valley vs Tulsa Roughnecks Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250041, 1468250041, 0, 0, 0, 0, 0, 0),
(25544, 25554, 'author', 'en', '[Live~HD] Orlando Pride vs Boston Breakers Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250064, 1468250064, 0, 0, 0, 0, 0, 0),
(25545, 25555, 'author', 'en', '[Live~HD] Deportivo La Guaira vs Llaneros de Guanare Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250081, 1468250081, 0, 0, 0, 0, 0, 0),
(25546, 25556, 'author', 'en', '[Live~HD] Monagas vs Deportivo Zulia Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468250098, 1468250098, 0, 0, 0, 0, 0, 0),
(25547, 25557, 'author', 'en', '[Live~HD] Zrinjski vs Legia Warszawa Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251167, 1468251167, 0, 0, 0, 0, 0, 0),
(25548, 25558, 'author', 'en', '[Live~HD] Qarabağ vs F91 Dudelange Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251199, 1468251199, 0, 0, 0, 0, 0, 0),
(25549, 25559, 'author', 'en', '[Live~HD] Dinamo Tbilisi vs Alashkert Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251222, 1468251222, 0, 0, 0, 0, 0, 0),
(25550, 25560, 'author', 'en', '[Live~HD] Hapoel Be''er Sheva vs Sheriff Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251252, 1468251252, 0, 0, 0, 0, 0, 0),
(25551, 25561, 'author', 'en', '[Live~HD] BATE vs SJK Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251273, 1468251273, 0, 0, 0, 0, 0, 0),
(25552, 25562, 'author', 'en', '[Live~HD] Lincoln Red Imps vs Celtic Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251293, 1468251293, 0, 0, 0, 0, 0, 0),
(25553, 25563, 'author', 'en', '[Live~HD] The New Saints vs APOEL Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251316, 1468251316, 0, 0, 0, 0, 0, 0),
(25554, 25564, 'author', 'en', '[Live~HD] Valletta vs Crvena Zvezda Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251344, 1468251344, 0, 0, 0, 0, 0, 0),
(25555, 25565, 'author', 'en', '[Live~HD] Wigry Suwałki vs AEK Athens Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251362, 1468251362, 0, 0, 0, 0, 0, 0),
(25556, 25566, 'author', 'en', '[Live~HD] Nantes vs Le Mont Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251388, 1468251388, 0, 0, 0, 0, 0, 0),
(25557, 25567, 'author', 'en', '[Live~HD] Thun vs Galatasaray Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251409, 1468251409, 0, 0, 0, 0, 0, 0),
(25558, 25568, 'author', 'en', '[Live~HD] Lille vs Red Star Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251426, 1468251426, 0, 0, 0, 0, 0, 0),
(25559, 25569, 'author', 'en', '[Live~HD] Caen vs Avranches Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251455, 1468251455, 0, 0, 0, 0, 0, 0),
(25560, 25570, 'author', 'en', '[Live~HD] FK Viktoria Plzen vs Fortuna Düsseldorf Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251471, 1468251471, 0, 0, 0, 0, 0, 0),
(25561, 25571, 'author', 'en', '[Live~HD] Altglienicke vs Werder Bremen Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251488, 1468251488, 0, 0, 0, 0, 0, 0),
(25562, 25572, 'author', 'en', '[Live~HD] Schackendorf vs Hamburger SV Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251513, 1468251513, 0, 0, 0, 0, 0, 0),
(25563, 25573, 'author', 'en', '[Live~HD] 4 De Abril vs Petro De Luanda Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251531, 1468251531, 0, 0, 0, 0, 0, 0),
(25564, 25574, 'author', 'en', '[Live~HD] Croatia vs Netherlands Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251633, 1468251633, 0, 0, 0, 0, 0, 0),
(25565, 25575, 'author', 'en', '[Live~HD] France vs England Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251649, 1468251649, 0, 0, 0, 0, 0, 0),
(25566, 25576, 'author', 'en', '[Live~HD] Atlantas vs Utenis Utena Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251670, 1468251670, 0, 0, 0, 0, 0, 0),
(25567, 25577, 'author', 'en', '[Live~HD] Forza FC vs Stallion Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251690, 1468251690, 0, 0, 0, 0, 0, 0),
(25568, 25578, 'author', 'en', '[Live~HD] Al Ittihad vs Al Wahda Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251729, 1468251729, 0, 0, 0, 0, 0, 0),
(25569, 25579, 'author', 'en', '[Live~HD] Al Mohafaza vs Jaish Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251758, 1468251758, 0, 0, 0, 0, 0, 0),
(25570, 25580, 'author', 'en', '[Live~HD] Karama vs Shorta Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251790, 1468251790, 0, 0, 0, 0, 0, 0),
(25571, 25581, 'author', 'en', '[Live~HD] Breidablik vs ÍA Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251806, 1468251806, 0, 0, 0, 0, 0, 0),
(25572, 25582, 'author', 'en', '[Live~HD] Stjarnan vs Fjölnir Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251823, 1468251823, 0, 0, 0, 0, 0, 0),
(25573, 25583, 'author', 'en', '[Live~HD] Thróttur Reykjavík vs Fylkir Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251840, 1468251840, 0, 0, 0, 0, 0, 0),
(25574, 25584, 'author', 'en', '[Live~HD] UTC Cajamarca vs Unión Comercio Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251858, 1468251858, 0, 0, 0, 0, 0, 0),
(25575, 25585, 'author', 'en', '[Live~HD] Cruzeiro vs Atlético PR Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251875, 1468251875, 0, 0, 0, 0, 0, 0),
(25576, 25586, 'author', 'en', '[Live~HD] Ukraine United vs Serbian White Eagles Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251918, 1468251918, 0, 0, 0, 0, 0, 0),
(25577, 25587, 'author', 'en', '[Live~HD] Águilas Doradas vs Atlético Nacional Live S-t.r.e.a.m.i.n-g Match', 1901, 1, 1468251942, 1468251942, 0, 0, 0, 0, 0, 0),
(25578, 25588, 'author', 'en', '[Live~HD] Deportes Quindío vs Deportivo Pereira Live S-t.r.e.a.m.i.n-g ', 1901, 1, 1468251986, 1468251986, 0, 0, 0, 0, 0, 0),
(25579, 25589, 'author', 'en', 'Official:~Pretty Little Liars Season 7 Episode 4 Episode 6 S tream', 1901, 1, 1468258030, 1468258030, 0, 0, 0, 0, 0, 0),
(25580, 25590, 'author', 'en', '[Live~HD] Pretty Little Liars Season 7 Episode 4 Episode 6 S tream', 1901, 1, 1468258493, 1468258493, 0, 0, 0, 0, 0, 0),
(25581, 25591, 'author', 'en', '[Live~HD] Masterchef Australia Season 8 Episode 53 S tream', 1901, 1, 1468258751, 1468258751, 0, 0, 0, 0, 0, 0),
(25582, 25592, 'author', 'en', '[Live~HD] Big Brother (UK) Season 17 Episode 36 S tream', 1901, 1, 1468259014, 1468259014, 0, 0, 0, 0, 0, 0),
(25583, 25593, 'author', 'en', '[TV.+]Groningen vs Queens Park Rangers Live Stream Online ', 2048, 1, 1468259695, 1468259695, 0, 0, 0, 0, 0, 0),
(25584, 25594, 'author', 'en', '[Live~HD] Wentworth Season 4 Episode 10 S tream', 1901, 1, 1468308096, 1468308096, 0, 0, 0, 0, 0, 0),
(25585, 25595, 'author', 'en', 'Lincocin Cure Menopause Purchase', 1888, 1, 1468308211, 1468308211, 0, 0, 0, 0, 0, 0),
(25586, 25596, 'author', 'en', 'Buy Fioricet Mastercard Legally', 1888, 1, 1468312483, 1468312483, 0, 0, 0, 0, 0, 0),
(25587, 25597, 'author', 'en', 'W atch Forza FC vs Stallion S treaming L ive O nline', 1820, 1, 1468315340, 1468315340, 0, 0, 0, 0, 0, 0),
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(25589, 25599, 'author', 'en', 'Soccer~:W atch Nantes vs Le Mont L ive S treaming Full Coverage...', 1820, 1, 1468315954, 1468315954, 0, 0, 0, 0, 0, 0),
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(25591, 25601, 'author', 'en', 'Soccer~:W atch Al Ittihad vs Al Wahda L ive S treaming Full Coverage...', 1820, 1, 1468315955, 1468315955, 0, 0, 0, 0, 0, 0),
(25592, 25602, 'author', 'en', 'Soccer~:W atch Croatia vs Netherlands L ive S treaming Full Coverage...', 1820, 1, 1468315955, 1468315955, 0, 0, 0, 0, 0, 0),
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(25638, 25648, 'author', 'en', 'Buy Rosuvastatin 200Mg', 1760, 1, 1468323330, 1468323330, 0, 0, 0, 0, 0, 0),
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(25753, 25763, 'author', 'en', 'Soccer~:W atch Ventforet Kofu vs Júbilo Iwata L ive S treaming Full Coverage...', 1820, 1, 1468402465, 1468402465, 0, 0, 0, 0, 0, 0),
(25754, 25764, 'author', 'en', 'Soccer~:W atch Jeonbuk Motors vs Bucheon 1995 L ive S treaming Full Coverage...', 1820, 1, 1468402467, 1468402467, 0, 0, 0, 0, 0, 0),
(25755, 25765, 'author', 'en', 'Soccer~:W atch Yokohama F. Marinos vs Vissel Kobe L ive S treaming Full Coverage...', 1820, 1, 1468402468, 1468402468, 0, 0, 0, 0, 0, 0),
(25756, 25766, 'author', 'en', 'Buy Brand Clotrimazole Lotrisone Amex', 1888, 1, 1468402493, 1468402493, 0, 0, 0, 0, 0, 0),
(25757, 25767, 'author', 'en', 'Soccer~:W atch Seoul vs Jeonnam Dragons L ive S treaming Full Coverage...', 1820, 1, 1468402797, 1468402797, 0, 0, 0, 0, 0, 0),
(25758, 25768, 'author', 'en', 'Soccer~:W atch Suwon Bluewings vs Seongnam L ive S treaming Full Coverage...', 1820, 1, 1468402799, 1468402799, 0, 0, 0, 0, 0, 0),
(25759, 25769, 'author', 'en', 'Soccer~:W atch Bulawayo FC vs Border Strikers L ive S treaming Full Coverage...', 1820, 1, 1468402801, 1468402801, 0, 0, 0, 0, 0, 0),
(25760, 25770, 'author', 'en', 'Soccer~:W atch Ulsan vs Incheon United L ive S treaming Full Coverage...', 1820, 1, 1468402801, 1468402801, 0, 0, 0, 0, 0, 0),
(25761, 25771, 'author', 'en', 'Soccer~:W atch Guangzhou R&F vs Hebei CFFC L ive S treaming Full Coverage...', 1820, 1, 1468402801, 1468402801, 0, 0, 0, 0, 0, 0),
(25762, 25772, 'author', 'en', 'Soccer~:W atch Chanthaburi vs Buriram United L ive S treaming Full Coverage...', 1820, 1, 1468402801, 1468402801, 0, 0, 0, 0, 0, 0),
(25763, 25773, 'author', 'en', 'Soccer~:W atch Chiangrai United vs Bangkok Glass L ive S treaming Full Coverage...', 1820, 1, 1468402802, 1468402802, 0, 0, 0, 0, 0, 0),
(25764, 25774, 'author', 'en', 'Soccer~:W atch Nonthaburi vs Samut Sakhon L ive S treaming Full Coverage...', 1820, 1, 1468402804, 1468402804, 0, 0, 0, 0, 0, 0),
(25765, 25775, 'author', 'en', 'Soccer~:W atch Port F.C. vs BEC Tero Sasana L ive S treaming Full Coverage...', 1820, 1, 1468402805, 1468402805, 0, 0, 0, 0, 0, 0),
(25766, 25776, 'author', 'en', 'Soccer~:W atch Port FC vs BEC Tero Sasana L ive S treaming Full Coverage...', 1820, 1, 1468402805, 1468402805, 0, 0, 0, 0, 0, 0),
(25767, 25777, 'author', 'en', 'Soccer~:W atch Prachuap vs Trat L ive S treaming Full Coverage...', 1820, 1, 1468402805, 1468402805, 0, 0, 0, 0, 0, 0),
(25768, 25778, 'author', 'en', 'Soccer~:W atch Sukhothai vs Samutprakarn L ive S treaming Full Coverage...', 1820, 1, 1468402806, 1468402806, 0, 0, 0, 0, 0, 0),
(25769, 25779, 'author', 'en', 'Soccer~:W atch Suphanburi vs Krung Thonburi L ive S treaming Full Coverage...', 1820, 1, 1468402808, 1468402808, 0, 0, 0, 0, 0, 0),
(25770, 25780, 'author', 'en', 'Soccer~:W atch Ubon Umt vs Ratchaburi L ive S treaming Full Coverage...', 1820, 1, 1468402808, 1468402808, 0, 0, 0, 0, 0, 0),
(25771, 25781, 'author', 'en', 'Soccer~:W atch Henan Jianye vs Jiangsu Sainty L ive S treaming Full Coverage...', 1820, 1, 1468403610, 1468403610, 0, 0, 0, 0, 0, 0),
(25772, 25782, 'author', 'en', 'Soccer~:W atch DPMM FC vs Warriors L ive S treaming Full Coverage...', 1820, 1, 1468403611, 1468403611, 0, 0, 0, 0, 0, 0),
(25773, 25783, 'author', 'en', 'Soccer~:W atch Tampines Rovers vs Hougang United L ive S treaming Full Coverage...', 1820, 1, 1468403612, 1468403612, 0, 0, 0, 0, 0, 0),
(25774, 25784, 'author', 'en', 'Soccer~:W atch Shandong Luneng vs Shijiazhuang Ever Bright L ive S treaming Full Coverage...', 1820, 1, 1468403612, 1468403612, 0, 0, 0, 0, 0, 0),
(25775, 25785, 'author', 'en', 'Soccer~:W atch Shanghai Shenhua vs Tianjin Songjiang L ive S treaming Full Coverage...', 1820, 1, 1468403613, 1468403613, 0, 0, 0, 0, 0, 0),
(25776, 25786, 'author', 'en', 'Soccer~:W atch Guangzhou Evergrande vs Beijing Guoan L ive S treaming Full Coverage...', 1820, 1, 1468403613, 1468403613, 0, 0, 0, 0, 0, 0),
(25777, 25787, 'author', 'en', 'Soccer~:W atch Army United vs Krabi L ive S treaming Full Coverage...', 1820, 1, 1468403615, 1468403615, 0, 0, 0, 0, 0, 0),
(25778, 25788, 'author', 'en', 'Soccer~:W atch Nakhon Ratchasima vs Nara United L ive S treaming Full Coverage...', 1820, 1, 1468403616, 1468403616, 0, 0, 0, 0, 0, 0),
(25779, 25789, 'author', 'en', 'Soccer~:W atch Chainat Hornbill vs Banbung United L ive S treaming Full Coverage...', 1820, 1, 1468403616, 1468403616, 0, 0, 0, 0, 0, 0),
(25780, 25790, 'author', 'en', 'Soccer~:W atch Chonburi vs Nong Bua Pitchaya L ive S treaming Full Coverage...', 1820, 1, 1468403617, 1468403617, 0, 0, 0, 0, 0, 0),
(25781, 25791, 'author', 'en', 'Soccer~:W atch Chonburi vs Nong Bua Pitchaya FC L ive S treaming Full Coverage...', 1820, 1, 1468403618, 1468403618, 0, 0, 0, 0, 0, 0),
(25782, 25792, 'author', 'en', 'Soccer~:W atch Khon Kaen United vs Chiangmai L ive S treaming Full Coverage...', 1820, 1, 1468403619, 1468403619, 0, 0, 0, 0, 0, 0),
(25783, 25793, 'author', 'en', 'Soccer~:W atch Pattaya United vs Muang Thong United L ive S treaming Full Coverage...', 1820, 1, 1468403619, 1468403619, 0, 0, 0, 0, 0, 0),
(25784, 25794, 'author', 'en', 'Soccer~:W atch Sisaket vs Songkhla United L ive S treaming Full Coverage...', 1820, 1, 1468403620, 1468403620, 0, 0, 0, 0, 0, 0),
(25785, 25795, 'author', 'en', 'Soccer~:W atch Chicken Inn vs Ngezi Platinum L ive S treaming Full Coverage...', 1820, 1, 1468403621, 1468403621, 0, 0, 0, 0, 0, 0),
(25786, 25796, 'author', 'en', 'Soccer~:W atch Everton vs Barnsley L ive S treaming Full Coverage...', 1820, 1, 1468403622, 1468403622, 0, 0, 0, 0, 0, 0),
(25787, 25797, 'author', 'en', 'Soccer~:W atch PSG vs West Bromwich Albion L ive S treaming Full Coverage...', 1820, 1, 1468403623, 1468403623, 0, 0, 0, 0, 0, 0),
(25788, 25798, 'author', 'en', 'Soccer~:W atch VPS vs PK-35 Vantaa L ive S treaming Full Coverage...', 1820, 1, 1468403626, 1468403626, 0, 0, 0, 0, 0, 0),
(25789, 25799, 'author', 'en', 'Soccer~:W atch MSV Duisburg vs Vysočina Jihlava L ive S treaming Full Coverage...', 1820, 1, 1468403626, 1468403626, 0, 0, 0, 0, 0, 0),
(25790, 25800, 'author', 'en', 'Soccer~:W atch Jablonec vs Kaiserslautern L ive S treaming Full Coverage...', 1820, 1, 1468403627, 1468403627, 0, 0, 0, 0, 0, 0),
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(25792, 25802, 'author', 'en', 'Soccer~:W atch Brentford vs Bochum L ive S treaming Full Coverage...', 1820, 1, 1468403629, 1468403629, 0, 0, 0, 0, 0, 0),
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(25794, 25804, 'author', 'en', 'Soccer~:W atch Basel vs Zenit L ive S treaming Full Coverage...', 1820, 1, 1468403630, 1468403630, 0, 0, 0, 0, 0, 0),
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(25796, 25806, 'author', 'en', 'Soccer~:W atch Vasalund vs Piteå L ive S treaming Full Coverage...', 1820, 1, 1468403631, 1468403631, 0, 0, 0, 0, 0, 0),
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(25798, 25808, 'author', 'en', 'Soccer~:W atch Rosenborg vs Norrköping L ive S treaming Full Coverage...', 1820, 1, 1468403634, 1468403634, 0, 0, 0, 0, 0, 0),
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(25803, 25813, 'author', 'en', 'Soccer~:W atch Fenerbahçe vs Panathinakos L ive S treaming Full Coverage...', 1820, 1, 1468403639, 1468403639, 0, 0, 0, 0, 0, 0),
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(25805, 25815, 'author', 'en', 'Buy Cod Diclofenac-Gel Voveran', 2062, 1, 1468408085, 1468408085, 0, 0, 0, 0, 0, 0),
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(25807, 25817, 'author', 'en', 'HD@!~TV Tampines Rovers vs Hougang United Live Streaming', 2049, 1, 1468413074, 1468413074, 0, 0, 0, 0, 0, 0),
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(25811, 25821, 'author', 'en', 'WATCH GAA@ Galway Vs Kilkenny live Leinster Hurling-Final TV', 2070, 1, 1468422402, 1468422402, 0, 0, 0, 0, 0, 0),
(25812, 25822, 'author', 'en', 'Vytorin Without Prescription Medications', 1760, 1, 1468424938, 1468424938, 0, 0, 0, 0, 0, 0),
(25813, 25823, 'author', 'en', 'dfsg', 1771, 1, 1468425330, 1468425330, 0, 0, 0, 0, 0, 0),
(25814, 25824, 'author', 'en', 'Javier', 1771, 1, 1468425471, 1468425471, 0, 0, 0, 0, 0, 0),
(25815, 25825, 'author', 'en', '✯{~❹❹❹~}✯ :: Pokemon hack android hack PokeBalls', 1771, 1, 1468425682, 1468425682, 0, 0, 0, 0, 0, 0),
(25816, 25826, 'author', 'en', '☂{{HACK}}☂ 13/07/2016 :: Pokemon cheats hacking hack PokeCoins, PokeBalls | Android', 1771, 1, 1468425703, 1468425703, 0, 0, 0, 0, 0, 0),
(25817, 25827, 'author', 'en', '◘{*{HACKPACK}*}◙ :: Pokemon mod generate PokeCoins,', 1771, 1, 1468425723, 1468425723, 0, 0, 0, 0, 0, 0),
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(25820, 25830, 'author', 'en', '❀{~GETHACK~}❃ 13-07-2016 :: Pokemon ipa hack ', 1771, 1, 1468425815, 1468425815, 0, 0, 0, 0, 0, 0),
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(25823, 25833, 'author', 'en', '◼[~[❻❻❻]~]◼ :: Pokemon hack ifile fRee PokeBalls | Android/iOS', 1771, 1, 1468425874, 1468425874, 0, 0, 0, 0, 0, 0),
(25824, 25834, 'author', 'en', '❖{~CHEAT~}❗ 13/07/2016 :: Pokemon cheats iphone ', 1771, 1, 1468425878, 1468425878, 0, 0, 0, 0, 0, 0),
(25825, 25835, 'author', 'en', '▲[!❷❷❷!]▲ :: Pokemon hack no Jailbreak ', 1771, 1, 1468425897, 1468425897, 0, 0, 0, 0, 0, 0),
(25826, 25836, 'author', 'en', '◢HACKGENERATOR 13-07-2016 :: Pokemon hack m;a;c; free PokeCoi.ns!, Po.keBal❒ls | And,roid', 1771, 1, 1468425943, 1468425943, 0, 0, 0, 0, 0, 0),
(25827, 25837, 'author', 'en', '☥[[❾❾❾]] :: Code de Triche Pokemon generate PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468425965, 1468425965, 0, 0, 0, 0, 0, 0),
(25828, 25838, 'author', 'en', '♳HACKTOOL♳ 1✩3-07-2016 :: Pokemon hack work PokeBalls | iOS-Andr☈oid', 1771, 1, 1468425988, 1468425988, 0, 0, 0, 0, 0, 0),
(25829, 25839, 'author', 'en', '❍{!hackgenerator!} 13/07/2016 :: Pokemon cheat tool PoKeCoins, PokeBalls | Android', 1771, 1, 1468425989, 1468425989, 0, 0, 0, 0, 0, 0),
(25830, 25840, 'author', 'en', '❞(*❼❼❼*) :: Pokemon cheats fo,r ipad ', 1771, 1, 1468426013, 1468426013, 0, 0, 0, 0, 0, 0),
(25831, 25841, 'author', 'en', '♺[*❷❷❷*]♺ :: Pokemon cheat tool PokeC.oins, PokeBal.ls | 2016', 1771, 1, 1468426036, 1468426036, 0, 0, 0, 0, 0, 0),
(25832, 25842, 'author', 'en', '◇{!HACKTOOL!} 13-07-2016 :: Pokemon hack jaibreak ', 1771, 1, 1468426088, 1468426088, 0, 0, 0, 0, 0, 0),
(25833, 25843, 'author', 'en', '◄{*{❷❷\\❷}*} :: Pokemon cheats that actually work ', 1771, 1, 1468426104, 1468426104, 0, 0, 0, 0, 0, 0),
(25834, 25844, 'author', 'en', '☗{*{july}*}☘ 13/07/2016 :: Pokemon APK IPA Mod PokeBalls | Android/iOS', 1771, 1, 1468426108, 1468426108, 0, 0, 0, 0, 0, 0),
(25835, 25845, 'author', 'en', '☏[~[MAY]~] :: Pokemon hAck codes hack PokeCoins, | Android', 1771, 1, 1468426111, 1468426111, 0, 0, 0, 0, 0, 0),
(25836, 25846, 'author', 'en', '✳#HACKENGINE :: Pokemon Juwelen Cheat haCk PokeCo.inS | iOS-AnDroid', 1771, 1, 1468426112, 1468426112, 0, 0, 0, 0, 0, 0),
(25837, 25847, 'author', 'en', '◜{{❶❶❶}}◝ :: Pokemon cheat review generate P♵okeCoins, PokeBalls', 1771, 1, 1468426113, 1468426113, 0, 0, 0, 0, 0, 0),
(25838, 25848, 'author', 'en', 'bigboss', 2068, 1, 1468426123, 1468426123, 0, 0, 0, 0, 0, 0),
(25839, 25849, 'author', 'en', '♬~!~❸❸❸♭ :: Pokemon hack mac hack Po♮keCoins, PokeBalls | Android', 1771, 1, 1468426126, 1468426126, 0, 0, 0, 0, 0, 0),
(25840, 25850, 'author', 'en', '☃[~[❼❼♱❼]~]☄ :: Pokemon cheats download hack PokeCoins, Poke|BAlls', 1771, 1, 1468426128, 1468426128, 0, 0, 0, 0, 0, 0),
(25841, 25851, 'author', 'en', '▴[![hackcheats]!]▵ 13.07.2016 :: Pokemon chea☫ts real free PokeCoins | Android/iOS', 1771, 1, 1468426130, 1468426130, 0, 0, 0, 0, 0, 0),
(25842, 25852, 'author', 'en', '▻{!{engine▰}!} :: Pokemon cheat real ', 1771, 1, 1468426134, 1468426134, 0, 0, 0, 0, 0, 0),
(25843, 25853, 'author', 'en', '▬(!(1◉3:07:2016)!)▭ :: cheat Pokemon free PokeCoins, PokeBalls | And☏roid', 1771, 1, 1468426134, 1468426134, 0, 0, 0, 0, 0, 0),
(25844, 25854, 'author', 'en', '◱(!(❹❹❹)!)◱ :: how to get hack tool Pokemon generate POkeBalls | Android', 1771, 1, 1468426149, 1468426149, 0, 0, 0, 0, 0, 0),
(25845, 25855, 'author', 'en', '▥(!(hackg☗enerator)!)▥ 13:07.:2016 :: Pokemon Triche free PokeBalls | 2016', 1771, 1, 1468426150, 1468426150, 0, 0, 0, 0, 0, 0),
(25846, 25856, 'author', 'en', '✍{!{❾❾❾}!} :: how to hack Pokemon generate PokeBalls', 1771, 1, 1468426155, 1468426155, 0, 0, 0, 0, 0, 0),
(25847, 25857, 'author', 'en', '▹(~HACKTOOL~)▹ 13:07:2✯016 :: Pokemon cheats f.or i*pad generate PokeBalls | Android/i◭OS', 1771, 1, 1468426157, 1468426157, 0, 0, 0, 0, 0, 0),
(25848, 25858, 'author', 'en', '✥❸❸❸ :: Pokemon hack online ', 1771, 1, 1468426159, 1468426159, 0, 0, 0, 0, 0, 0),
(25849, 25859, 'author', 'en', '☨{!❶❶❶!} :: Pokemon hack activation code free PokeCoins | 2016', 1771, 1, 1468426173, 1468426173, 0, 0, 0, 0, 0, 0),
(25850, 25860, 'author', 'en', '✧(!(❻❻❻)!)✧ :: Pokemon Hacken P.okeCoins | iOS.-Android', 1771, 1, 1468426173, 1468426173, 0, 0, 0, 0, 0, 0),
(25851, 25861, 'author', 'en', '▣|_/¯❺❺❺¯\\_|▤ :: Pokemon hAck mac Po/♘keCoins, PokeBalls || Android/iOS', 1771, 1, 1468426177, 1468426177, 0, 0, 0, 0, 0, 0),
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(25853, 25863, 'author', 'en', '☿_/¯generator¯\\_ 13:07:2016 :: Pokemon hack mod ap☨k hack PokeBalls | iO◿S-♑Android', 1771, 1, 1468426181, 1468426181, 0, 0, 0, 0, 0, 0),
(25854, 25864, 'author', 'en', '♾(!❷❷❷!) :: Pokemon apk cheats Free PokeCoins | iOS-Android', 1771, 1, 1468426193, 1468426193, 0, 0, 0, 0, 0, 0),
(25855, 25865, 'author', 'en', '♶$MOD♶ ::□ Astuces Pour Pokemon ', 1771, 1, 1468426195, 1468426195, 0, 0, 0, 0, 0, 0),
(25856, 25866, 'author', 'en', '♈¯\\_HACKPACK_/¯♈ 13.07.2♘016 :: Pokemon Hack No Surv;eY generate PokeCoins, PokeBalls | Android', 1771, 1, 1468426198, 1468426198, 0, 0, 0, 0, 0, 0),
(25857, 25867, 'author', 'en', '✍|_/¯❺❺♈❺¯\\_|✂ :: Pokemon hack no download ✉ g.e.n.e.r.a.t.e. PokeCoins | 2016', 1771, 1, 1468426201, 1468426201, 0, 0, 0, 0, 0, 0),
(25858, 25868, 'author', 'en', '☐megahack 13/07/2016 :: Pokemon Hack i◀Phone iPad Android generate PokeCo■ins, PokeBalls | iOS-An☝droid', 1771, 1, 1468426228, 1468426228, 0, 0, 0, 0, 0, 0),
(25859, 25869, 'author', 'en', '◄$HACK 13/07/2016 :: Pokemon hack and cheats free PokeCoins, PokeBalls | ANdroid', 1771, 1, 1468426246, 1468426246, 0, 0, 0, 0, 0, 0),
(25860, 25870, 'author', 'en', '▻(!❺❺❺!)▻ :: Pokemon hack telechar✡ger✲ ', 1771, 1, 1468426247, 1468426247, 0, 0, 0, 0, 0, 0),
(25861, 25871, 'author', 'en', '♝[[❻❻❻]] :: how to hAck Pokemon free PokeC☵o.ins | AndroiD', 1771, 1, 1468426253, 1468426253, 0, 0, 0, 0, 0, 0),
(25862, 25872, 'author', 'en', 'DIRECT..[##]John Lineker vs Michael McDonald L.i.v.e  S.t.r.e.a.m Main Event on Pc,Tv,Macbook', 2068, 1, 1468426260, 1468426260, 0, 0, 0, 0, 0, 0),
(25863, 25873, 'author', 'en', '✡{*�..��❷❷*} :: Pokemon hack nO jailbreak generate PokeCoins, PokeBalls | iOS-Andr.oiD', 1771, 1, 1468426268, 1468426268, 0, 0, 0, 0, 0, 0),
(25864, 25874, 'author', 'en', '❆((❼❼❼))❆ :: Pokemon Cheats ', 1771, 1, 1468426292, 1468426292, 0, 0, 0, 0, 0, 0),
(25865, 25875, 'author', 'en', '☆{!{07-13-2.016}!} :: Pokemon cheat shEet hack PokeCoiNs | Android/iOS', 1771, 1, 1468426292, 1468426292, 0, 0, 0, 0, 0, 0),
(25866, 25876, 'author', 'en', '◿{*mod*}☠ :♁: Pokemon hack Android no root PokeCoins', 1771, 1, 1468426296, 1468426296, 0, 0, 0, 0, 0, 0),
(25867, 25877, 'author', 'en', '✈{~{❼❼❼}~}⚓ :: Pokemon ti▿ps for beginnErs ', 1771, 1, 1468426298, 1468426298, 0, 0, 0, 0, 0, 0),
(25868, 25878, 'author', 'en', '✥MAY✦ 13.07.2016 :: Pokemon Hack jaibreak ', 1771, 1, 1468426310, 1468426310, 0, 0, 0, 0, 0, 0),
(25869, 25879, 'author', 'en', '♝❽,❽❽♝ :!: Pokemon Pirater hack PokeCoIns, PokeBalls', 1771, 1, 1468426325, 1468426325, 0, 0, 0, 0, 0, 0),
(25870, 25880, 'author', 'en', '▥�:��❶❶▥ :: Pokemon hack ifunbox ', 1771, 1, 1468426327, 1468426327, 0, 0, 0, 0, 0, 0),
(25871, 25881, 'author', 'en', '◚{*❼❼❼*}◚ :: Pokemon cheats tha.t work ', 1771, 1, 1468426333, 1468426333, 0, 0, 0, 0, 0, 0),
(25872, 25882, 'author', 'en', '◷~!~CHEAT◸ :: Pokemon hack for android ', 1771, 1, 1468426335, 1468426335, 0, 0, 0, 0, 0, 0),
(25873, 25883, 'author', 'en', '☪!❹♃❹❹☫ :: Pokemon cheat apk hack PokeBalls', 1771, 1, 1468426343, 1468426343, 0, 0, 0, 0, 0, 0),
(25874, 25884, 'author', 'en', '♾[~[❺❺❺]~]♾ :: Pokemon apk cheat.s free PokeCoins | Android/iOS', 1771, 1, 1468426353, 1468426353, 0, 0, 0, 0, 0, 0),
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(25876, 25886, 'author', 'en', '❧(❷❷❷) :: Pokemon tips for noobs hack PokeBall❚s | Android/iOS', 1771, 1, 1468426358, 1468426358, 0, 0, 0, 0, 0, 0),
(25877, 25887, 'author', 'en', '☤[!�^��❾❾!]☥ :: Pokemon hack software ', 1771, 1, 1468426361, 1468426361, 0, 0, 0, 0, 0, 0),
(25878, 25888, 'author', 'en', '◘(*(❹❹❹)*)◘ :: Pokemon hack that works free Poke♆Balls | 2016', 1771, 1, 1468426364, 1468426364, 0, 0, 0, 0, 0, 0),
(25879, 25889, 'author', 'en', '♽(~(�\\�\\�\\�\\�\\�\\�\\�\\�\\)~)♽ :: Pokemon hack for android PokeCoins | iOS-Android', 1771, 1, 1468426375, 1468426375, 0, 0, 0, 0, 0, 0),
(25880, 25890, 'author', 'en', '♍❻❻❻♎ :: Pokemon hack online ', 1771, 1, 1468426378, 1468426378, 0, 0, 0, 0, 0, 0),
(25881, 25891, 'author', 'en', '◁_/¯�❏��❸❸¯\\_◂ :: Pokemon cheat app PokeCoins, PokeBalls', 1771, 1, 1468426380, 1468426380, 0, 0, 0, 0, 0, 0),
(25882, 25892, 'author', 'en', '◾[!gethack!]◿ 13/07/2016 :: Pokemon cheat tool PokeCoins | iOS-Android', 1771, 1, 1468426381, 1468426381, 0, 0, 0, 0, 0, 0),
(25883, 25893, 'author', 'en', '✪$❺❺❺ :: Pokemon hack tool online hack PokeBalls | Android', 1771, 1, 1468426403, 1468426403, 0, 0, 0, 0, 0, 0),
(25884, 25894, 'author', 'en', '♐{!FULLHACK!}♐ 13.07.2016 :: Pokemon HaCk iP❃hone iPad A.ndroid hack PokeBalls! | 2016', 1771, 1, 1468426408, 1468426408, 0, 0, 0, 0, 0, 0),
(25885, 25895, 'author', 'en', '☆❹❹❹☆ :: free game hack tools Pokemon PokeCoins, PokEBalls | A◦ndroid/iOS', 1771, 1, 1468426419, 1468426419, 0, 0, 0, 0, 0, 0),
(25886, 25896, 'author', 'en', '◤|_/¯❼❼❼¯\\_|◤ :: Astuces. Pour Pokemon PokeBalls | 2016', 1771, 1, 1468426422, 1468426422, 0, 0, 0, 0, 0, 0),
(25887, 25897, 'author', 'en', '♕[!❾❾❾..!] :: Pokemon cheats reviews free PokeCoins | Android/iOS', 1771, 1, 1468426425, 1468426425, 0, 0, 0, 0, 0, 0),
(25888, 25898, 'author', 'en', '✂(HACKED)✂ 13:07:2016 ::☆ Pokemon cheat Download ', 1771, 1, 1468426429, 1468426429, 0, 0, 0, 0, 0, 0),
(25889, 25899, 'author', 'en', '◶❹❹❹◶ :: Pokemon Juwelen CHeat free PokeCoins | Android/iOS', 1771, 1, 1468426440, 1468426440, 0, 0, 0, 0, 0, 0),
(25890, 25900, 'author', 'en', '☱❷❷❷☱ :: Pokemon hack proof hack PokeBalls | iOS-Android', 1771, 1, 1468426443, 1468426443, 0, 0, 0, 0, 0, 0),
(25891, 25901, 'author', 'en', '◜[!❶❶❶!]◜ :: Pokemon Hacken ', 1771, 1, 1468426445, 1468426445, 0, 0, 0, 0, 0, 0),
(25892, 25902, 'author', 'en', '▤❸❸❸▤ :: Pokemon hack no download hack PokeCoins, PokeBalls', 1771, 1, 1468426449, 1468426449, 0, 0, 0, 0, 0, 0),
(25893, 25903, 'author', 'en', '♈((GEN☣ERAT✫OR))♈ 13:07:2016 :: Pokemon hacked apk ', 1771, 1, 1468426450, 1468426450, 0, 0, 0, 0, 0, 0),
(25894, 25904, 'author', 'en', '♺(*(❹❹❹)*) :: Pokemon Hack No Survey free PokeBal✬ls | 2☾016', 1771, 1, 1468426460, 1468426460, 0, 0, 0, 0, 0, 0),
(25895, 25905, 'author', 'en', '❕{~{❺♆❺❺}~} :: Pokemon hack activation code hack PokeCoins, PokeBalls | Android', 1771, 1, 1468426465, 1468426465, 0, 0, 0, 0, 0, 0),
(25896, 25906, 'author', 'en', '♅¯\\_HACKPACK_/¯♅ 13:07::2016 :: Pokemon Glitch PokeCoins, PokeBalls | Android♤/iOS', 1771, 1, 1468426466, 1468426466, 0, 0, 0, 0, 0, 0),
(25897, 25907, 'author', 'en', '♏((❶❶❶))♏ :: Pokemon hack tool online. hack PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468426471, 1468426471, 0, 0, 0, 0, 0, 0),
(25898, 25908, 'author', 'en', '♛{!❼❼❼!}♛ :: Pokemon APK IPA Mod g☠enerate Pok✨eBalls', 1771, 1, 1468426473, 1468426473, 0, 0, 0, 0, 0, 0),
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(25900, 25910, 'author', 'en', '☮(*(C☰HEATS)*)☰ 13/07/2016 :: Pokemon cheats for mobile generate Pok..eBalls | Androi.d/iOS', 1771, 1, 1468426494, 1468426494, 0, 0, 0, 0, 0, 0),
(25901, 25911, 'author', 'en', '™{~❾❾❾~}™ :: Pokemon Glitch and Glitches generate PokeCoins, PokeBalls | 2016', 1771, 1, 1468426496, 1468426496, 0, 0, 0, 0, 0, 0),
(25902, 25912, 'author', 'en', '♈{~{hack}~}♈ 13:07:2016 :: Telecharger Pokemon H/a/c/k/ GRatuit ', 1771, 1, 1468426496, 1468426496, 0, 0, 0, 0, 0, 0),
(25903, 25913, 'author', 'en', '◗{*ENGINE*}◗ 13.07.◙2016 :: Pokemon hack no download hack PokeCoins | Android/iOS', 1771, 1, 1468426496, 1468426496, 0, 0, 0, 0, 0, 0),
(25904, 25914, 'author', 'en', '◃[![�.�.�.�.�.�.�.�.�.]!]◃ :: Pokemon hack no jailbreak PokeCoins, PokeBalls | Android', 1771, 1, 1468426516, 1468426516, 0, 0, 0, 0, 0, 0),
(25905, 25915, 'author', 'en', '◜{{❷❷�✭�}}◝ :: Pokemon cheaTs hacks hack PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468426517, 1468426517, 0, 0, 0, 0, 0, 0),
(25906, 25916, 'author', 'en', '▮#❼❼❼▮ :: Pokemon cheat tool genErate PokeCoins, PoKe*Balls', 1771, 1, 1468426519, 1468426519, 0, 0, 0, 0, 0, 0),
(25907, 25917, 'author', 'en', '✱~!~HACKENGINE✲ 13/07/2016 :: Pokemon Online Generator fre▨e PokeCoins', 1771, 1, 1468426520, 1468426520, 0, 0, 0, 0, 0, 0),
(25908, 25918, 'author', 'en', '●[❼❼❼]◐ :: Pokemon cheat tool ', 1771, 1, 1468426536, 1468426536, 0, 0, 0, 0, 0, 0),
(25909, 25919, 'author', 'en', '◑{!{HACKGENERATOR}!}◑ 13:07:2016 :: Pokemon hack generato;r PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468426541, 1468426541, 0, 0, 0, 0, 0, 0),
(25910, 25920, 'author', 'en', '▭¯\\_13.07.2016_/¯▭ :: free Game ha◲Ck tool♴s Pokemon free PokeCoins, Po™keBalls', 1771, 1, 1468426561, 1468426561, 0, 0, 0, 0, 0, 0),
(25911, 25921, 'author', 'en', '♁{*❻❻�*�*}♂ :: Pokemon hack ios f.ree PokeCoins, PokeBalls | Android▤', 1771, 1, 1468426565, 1468426565, 0, 0, 0, 0, 0, 0),
(25912, 25922, 'author', 'en', '◘{!{❺❺❺}!}◘ :: Pokemon Hack Gratis ', 1771, 1, 1468426565, 1468426565, 0, 0, 0, 0, 0, 0),
(25913, 25923, 'author', 'en', '♇[!HACKCHEATS!] :: Pokemon cheats that a\\ctu|ally work ', 1771, 1, 1468426565, 1468426565, 0, 0, 0, 0, 0, 0),
(25914, 25924, 'author', 'en', '♿[~C.H.E.A.T.~] 13♺-07-2016 :: Pokemon Gl❟itch ', 1771, 1, 1468426583, 1468426583, 0, 0, 0, 0, 0, 0),
(25915, 25925, 'author', 'en', '☚(!❾◾�☫�❾!) :: Pokemon Triche free PokeCoins | iOS-Android', 1771, 1, 1468426586, 1468426586, 0, 0, 0, 0, 0, 0),
(25916, 25926, 'author', 'en', '☓[!�*��❺♎❺!]☓ :: Pokemon tips for beginners ', 1771, 1, 1468426588, 1468426588, 0, 0, 0, 0, 0, 0),
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(25918, 25928, 'author', 'en', '◽~!~�/��❷❷◾ :: Pokemon che✫ats tha,t work ', 1771, 1, 1468426591, 1468426591, 0, 0, 0, 0, 0, 0),
(25919, 25929, 'author', 'en', '◅(GENERATOR)◆ 13.07.2016 :: Pokemon cheats for iphone hack PokeBalls', 1771, 1, 1468426608, 1468426608, 0, 0, 0, 0, 0, 0),
(25920, 25930, 'author', 'en', '♽{*❻❻❻*}♾ :: Pokemon hack ifunbo▮x ', 1771, 1, 1468426609, 1468426609, 0, 0, 0, 0, 0, 0),
(25921, 25931, 'author', 'en', '♛|¯\\_❾❾❾_/¯| :: free game hack tools Pokemon PokeCoins, PokeBalls', 1771, 1, 1468426611, 1468426611, 0, 0, 0, 0, 0, 0),
(25922, 25932, 'author', 'en', '▦{!{❹❹❹}!}▧ :: Pokemon Cheats ', 1771, 1, 1468426612, 1468426612, 0, 0, 0, 0, 0, 0),
(25923, 25933, 'author', 'en', '♺_/¯❷❷❷¯\\_ :: Pokemon hack no d™ownload  ', 1771, 1, 1468426614, 1468426614, 0, 0, 0, 0, 0, 0),
(25924, 25934, 'author', 'en', '◶[![�♟��❹❹]!] :: Pokemon a.p.k. cheats free P.o.k.e.C.o.i.n.s. | iOS-Android', 1771, 1, 1468426630, 1468426630, 0, 0, 0, 0, 0, 0),
(25925, 25935, 'author', 'en', '♲!❽❽❽♳ :.:. Pokemon Tips and Tricks hack PokeCoins | 2016', 1771, 1, 1468426631, 1468426631, 0, 0, 0, 0, 0, 0),
(25926, 25936, 'author', 'en', '▻[*[07/13/2016]*]▻ :: Pokemon Hack Gratis ', 1771, 1, 1468426633, 1468426633, 0, 0, 0, 0, 0, 0),
(25927, 25937, 'author', 'en', '☯[![�.��❻❻]!]✈ :: Pokemon cheat that works hack PokeBaLls | Android', 1771, 1, 1468426635, 1468426635, 0, 0, 0, 0, 0, 0),
(25928, 25938, 'author', 'en', '☝[~❶❓❶❶~]☝ :: Pokemon cheat app PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468426639, 1468426639, 0, 0, 0, 0, 0, 0),
(25929, 25939, 'author', 'en', '△(!(❷❷❷)!)▴ :: Pokemon cheat that worKs free PokeBalls | iOS-Android', 1771, 1, 1468426655, 1468426655, 0, 0, 0, 0, 0, 0),
(25930, 25940, 'author', 'en', '❒{~{MOD;}~}❒ 13◝..07.2016 :: Pokemon cheat do♉wnload hack PokEBalls | Android/iOS', 1771, 1, 1468426658, 1468426658, 0, 0, 0, 0, 0, 0),
(25931, 25941, 'author', 'en', '♡{*{HA\\CKTOOL}*}♢ :: Pokemon hack for android Po,keCoins', 1771, 1, 1468426658, 1468426658, 0, 0, 0, 0, 0, 0),
(25932, 25942, 'author', 'en', '☗#�\\�\\�\\�\\�\\�\\�\\�\\�\\ :: Pokemon chea☨ts free free PokeBalls | i!OS-◪Android', 1771, 1, 1468426667, 1468426667, 0, 0, 0, 0, 0, 0),
(25933, 25943, 'author', 'en', '✪¯\\_2016_/¯✪ 13:07:2016 :: Pokemon hack for iphone generate PokeCoins | Android/iOS', 1771, 1, 1468426693, 1468426693, 0, 0, 0, 0, 0, 0),
(25934, 25944, 'author', 'en', '▤[*[❺❺❺]*]▤ :: Pokemon Juwelen Hack generate PokeCoin❞s | Android/iOS', 1771, 1, 1468426702, 1468426702, 0, 0, 0, 0, 0, 0),
(25935, 25945, 'author', 'en', '♐❺�▭��❺ :: Pokemon hack tool online ', 1771, 1, 1468426719, 1468426719, 0, 0, 0, 0, 0, 0),
(25936, 25946, 'author', 'en', '◼{*{ONLINE}*}◽ :: Pokemon hack jaibreak PokeCoins | Android:', 1771, 1, 1468426724, 1468426724, 0, 0, 0, 0, 0, 0),
(25937, 25947, 'author', 'en', '♯_/¯07/13./2016¯\\_♯ :: Pokemon Mod Apk ', 1771, 1, 1468426727, 1468426727, 0, 0, 0, 0, 0, 0),
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(25940, 25950, 'author', 'en', '✣{!❶❶❶!} :: Pokemon hack jaibreak genera.te P..o..k..e..C..o..i..n..s.. | Android/iOS', 1771, 1, 1468426746, 1468426746, 0, 0, 0, 0, 0, 0),
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(25942, 25952, 'author', 'en', '✼[*[FULLHACK]*]✽ 13-07|-2016 :: Pokemon H♶ack Cheats Trainer ', 1771, 1, 1468426750, 1468426750, 0, 0, 0, 0, 0, 0),
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(25946, 25956, 'author', 'en', '▮❹❹.❹▮ :: Pokemon hack activation code free PokeCoins, PokeBalls | Andr,oid/iOS', 1771, 1, 1468426771, 1468426771, 0, 0, 0, 0, 0, 0),
(25947, 25957, 'author', 'en', '☙$�^��❼.❼☙ :: Pokemon hack safe hack PokeB☕alls | Android', 1771, 1, 1468426774, 1468426774, 0, 0, 0, 0, 0, 0),
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(25949, 25959, 'author', 'en', '▦(h✬ack)▦ 13-07-2016 :: Pokemon cheat that works free PokeBalls | Android/iOS', 1771, 1, 1468426786, 1468426786, 0, 0, 0, 0, 0, 0),
(25950, 25960, 'author', 'en', '♾(�.��☲�,��❺)♾ :: Pokemon hack d☧ownload free PokeBalls', 1771, 1, 1468426789, 1468426789, 0, 0, 0, 0, 0, 0),
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(25952, 25962, 'author', 'en', '♛{~{ONLINE}~} 13-07-2016 :: Pokemon Hack 2016 hack PokeBalls..', 1771, 1, 1468426795, 1468426795, 0, 0, 0, 0, 0, 0),
(25953, 25963, 'author', 'en', '☄{hacke◍d}☇ 13/07/2016 :: Pokemon c.heats anD hacks hack PokeBalls | 201;6', 1771, 1, 1468426800, 1468426800, 0, 0, 0, 0, 0, 0),
(25954, 25964, 'author', 'en', '✺(!❺❺❺!)✺ :: Pokemon Hack Po|keCoins, PokeBalls | iOS-Android', 1771, 1, 1468426810, 1468426810, 0, 0, 0, 0, 0, 0),
(25955, 25965, 'author', 'en', '♉_/¯❾❾❾¯\\_♉ ::♙ Code de Triche Pokemon ^', 1771, 1, 1468426812, 1468426812, 0, 0, 0, 0, 0, 0),
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(25962, 25972, 'author', 'en', '✽(2016) 13/07/2016 :: Pokemon APK I♨PA MoD ', 1771, 1, 1468426872, 1468426872, 0, 0, 0, 0, 0, 0),
(25963, 25973, 'author', 'en', '▾(!gethack!)▿ 13.07.2016 :: Pokemon Astuce PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468426872, 1468426872, 0, 0, 0, 0, 0, 0),
(25964, 25974, 'author', 'en', '❑(!❸❸❸!) :: Pokemon pi◵raT!er telecharger generate PokeBa.lls | Android/iOS', 1771, 1, 1468426881, 1468426881, 0, 0, 0, 0, 0, 0),
(25965, 25975, 'author', 'en', '☃(*(❽❽❽)*)☃ :: Pokemon hack no downLo*ad  hack PokeCoins, PokeBalls | Android', 1771, 1, 1468426898, 1468426898, 0, 0, 0, 0, 0, 0),
(25966, 25976, 'author', 'en', '♊gethack 13:07:2016 :: Pokemon Online Hack frEe PokeBalls | Android/iOS', 1771, 1, 1468426901, 1468426901, 0, 0, 0, 0, 0, 0),
(25967, 25977, 'author', 'en', '◀{~�/��❻❻~}◀ :: Pokemon Hacker hack PokeCoins | 2016', 1771, 1, 1468426901, 1468426901, 0, 0, 0, 0, 0, 0),
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(25972, 25982, 'author', 'en', '☲HACKCHEATS :: Pokemon hack i,file hack PokeCoins | Android/iOS', 1771, 1, 1468426949, 1468426949, 0, 0, 0, 0, 0, 0),
(25973, 25983, 'author', 'en', '☌[~ME◈GAHACK~]☌ 1/3/./0/7/./2/0/1/6/ :: Pokemon Online Generator hack PokeBalls', 1771, 1, 1468426952, 1468426952, 0, 0, 0, 0, 0, 0),
(25974, 25984, 'author', 'en', '♥!2016♥ 13:07:2016 :: Pokemon ch.eat that wor▶ks Poke♿CoiNs, PokeBalls | Android/iOS', 1771, 1, 1468426954, 1468426954, 0, 0, 0, 0, 0, 0),
(25975, 25985, 'author', 'en', '♿❷❷❷♀ :: Pokemon Hack No Root free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468426970, 1468426970, 0, 0, 0, 0, 0, 0),
(25976, 25986, 'author', 'en', '♌(!(july)!)♌ 13-07-2016 :: Pokemon hack ipHone☴ no root hack PokeCoins | AndrOid', 1771, 1, 1468426974, 1468426974, 0, 0, 0, 0, 0, 0),
(25977, 25987, 'author', 'en', '♓_/¯❸❸❸¯\\_♔ :: how tO get hack tool✤ Pokemon hac|k PokeCoins, PokeBalls | 2016', 1771, 1, 1468426975, 1468426975, 0, 0, 0, 0, 0, 0),
(25978, 25988, 'author', 'en', '▦(*(hackcheats)*)▧ 13/07/2016 :: Pokemon cheat engine .', 1771, 1, 1468426975, 1468426975, 0, 0, 0, 0, 0, 0),
(25979, 25989, 'author', 'en', '☞###HACKPACK☟ 13-07-2016 :: Pokemon A.P.K. IPA Mod ', 1771, 1, 1468426991, 1468426991, 0, 0, 0, 0, 0, 0),
(25980, 25990, 'author', 'en', '®[*❶❶❶*] :: A.s.t.u.c.e.s. Pour Pokemon ', 1771, 1, 1468426995, 1468426995, 0, 0, 0, 0, 0, 0),
(25981, 25991, 'author', 'en', '♉_/¯❾❾�.�¯\\_♉ :: Pokemon hack andRoid ', 1771, 1, 1468427020, 1468427020, 0, 0, 0, 0, 0, 0),
(25982, 25992, 'author', 'en', '☙(!❶❶❶!) :: Pokemon Juwelen CheaT generate PokeBalls | Android', 1771, 1, 1468427021, 1468427021, 0, 0, 0, 0, 0, 0),
(25983, 25993, 'author', 'en', '◻(*(onli☢ne)*)◻ :: Pokemon cheat codes PokeBalls | 2016', 1771, 1, 1468427022, 1468427022, 0, 0, 0, 0, 0, 0),
(25984, 25994, 'author', 'en', '✸[![❺❺❺]!]✸ :: Pokemon Juwelen Cheat ', 1771, 1, 1468427022, 1468427022, 0, 0, 0, 0, 0, 0),
(25985, 25995, 'author', 'en', '▼{!{❸❸❸}!}▼ :: Pokemon cheat sheet generate PokeBalls | And*roid/iOS', 1771, 1, 1468427044, 1468427044, 0, 0, 0, 0, 0, 0),
(25986, 25996, 'author', 'en', '►[!2016!]▻ :: how tO get hack tool Pokemon PokeCOi.ns, PokeBalls | Android/iOS', 1771, 1, 1468427047, 1468427047, 0, 0, 0, 0, 0, 0),
(25987, 25997, 'author', 'en', '♵~!~CHEATS♶ 13-07-2016 :: Pokemon Triche ', 1771, 1, 1468427049, 1468427049, 0, 0, 0, 0, 0, 0),
(25988, 25998, 'author', 'en', '♹(!(❸❸❸)!) :: Pokemon hack iPad PokeCoins | A.nd♣roid/iOS', 1771, 1, 1468427071, 1468427071, 0, 0, 0, 0, 0, 0),
(25989, 25999, 'author', 'en', '♳[~[MAY]~]♴ :: Pokemon hack iphone no root generate PokeBalls | iOS-Android', 1771, 1, 1468427072, 1468427072, 0, 0, 0, 0, 0, 0),
(25990, 26000, 'author', 'en', '☪[![❼❼❼]!]☫ :: Pokemon hack apk generate Pok:eBalls | Android', 1771, 1, 1468427085, 1468427085, 0, 0, 0, 0, 0, 0),
(25991, 26001, 'author', 'en', '❁[![MOD]!] :: Pokemon Mod Apk ', 1771, 1, 1468427087, 1468427087, 0, 0, 0, 0, 0, 0),
(25992, 26002, 'author', 'en', '◿❺❺❺ :: Pokemon cheats download free PokeCoins | 2016', 1771, 1, 1468427091, 1468427091, 0, 0, 0, 0, 0, 0),
(25993, 26003, 'author', 'en', '◵{!�◐��❹❹!}◵ :: Pokemon Hack iPhone iPad Android hack PokeCoins, PokeBalls', 1771, 1, 1468427093, 1468427093, 0, 0, 0, 0, 0, 0),
(25994, 26004, 'author', 'en', '◿[![❷�.�❷]!] :: Pokemon tips cheats generate PokeBalls | iOS-Android', 1771, 1, 1468427107, 1468427107, 0, 0, 0, 0, 0, 0),
(25995, 26005, 'author', 'en', '☇$GENERAT☉OR 13.07.2016 :: Pokemon Hack Cheat Juwelen Deutsch! ', 1771, 1, 1468427108, 1468427108, 0, 0, 0, 0, 0, 0),
(25996, 26006, 'author', 'en', '◺{*{❶❶❶}*}◻ :: Pokemon cheat review. generate PokeCoins, PokeBalls', 1771, 1, 1468427110, 1468427110, 0, 0, 0, 0, 0, 0),
(25997, 26007, 'author', 'en', '♉(~❽�..�❽~)♉ :: Pokemon hack t.o.o.l. online f.r.e.e. Poke♉Coins | iOS-Android', 1771, 1, 1468427113, 1468427113, 0, 0, 0, 0, 0, 0),
(25998, 26008, 'author', 'en', '☥(!CHEAT!)☦ 13:07◇:2016 :: Pokemon hack no root PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468427115, 1468427115, 0, 0, 0, 0, 0, 0),
(25999, 26009, 'author', 'en', '▱{~❹❹❹~}▲ :: Pokemon hack cheats ', 1771, 1, 1468427128, 1468427128, 0, 0, 0, 0, 0, 0),
(26000, 26010, 'author', 'en', '☯(!❼❼❼!)☯ :: Pokemon Hack iPho❀ne iPad And♚roid ', 1771, 1, 1468427133, 1468427133, 0, 0, 0, 0, 0, 0),
(26001, 26011, 'author', 'en', '☘(*(❺❺❺)*) :: Pokemon Juwelen Hack ', 1771, 1, 1468427136, 1468427136, 0, 0, 0, 0, 0, 0),
(26002, 26012, 'author', 'en', '✫hackcheat.s 13.07.2016 :: Pokemon Hack Tool APK IPA ', 1771, 1, 1468427136, 1468427136, 0, 0, 0, 0, 0, 0),
(26003, 26013, 'author', 'en', '▪¯\\_GETHACK_/¯▪ :: Pokemon hack telecharger generate PokeBalls | 2016', 1771, 1, 1468427148, 1468427148, 0, 0, 0, 0, 0, 0),
(26004, 26014, 'author', 'en', '♘!❹❹❹♘ :: Pokemon tips for noobs PokeCoins, POkeBalls | Android', 1771, 1, 1468427154, 1468427154, 0, 0, 0, 0, 0, 0),
(26005, 26015, 'author', 'en', '◁❻❻❻◁ :: Pokemon tips cheats ge❃nerate PokeCoins, PokeBalls | And,roid/iOS', 1771, 1, 1468427157, 1468427157, 0, 0, 0, 0, 0, 0),
(26006, 26016, 'author', 'en', '▧❾❾❾▧ :: Pokemon cheats iphone hack PokeCoins | iOS-Android', 1771, 1, 1468427159, 1468427159, 0, 0, 0, 0, 0, 0),
(26007, 26017, 'author', 'en', '◲¯\\_❶❶❶_/¯◳ :: Pokemon haCk ifun*box hack PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427159, 1468427159, 0, 0, 0, 0, 0, 0),
(26008, 26018, 'author', 'en', '♡[![mod]!]♡ :: Pokemon Glitch and Glitches ', 1771, 1, 1468427175, 1468427175, 0, 0, 0, 0, 0, 0),
(26009, 26019, 'author', 'en', '♳(*(hacked)*)♳ 13/0:7/2016 :: Pokemon hack real PokeBalls | Android/iOS', 1771, 1, 1468427181, 1468427181, 0, 0, 0, 0, 0, 0),
(26010, 26020, 'author', 'en', '☸(!❶❶❶!) :: Pokemon hack android no root ', 1771, 1, 1468427191, 1468427191, 0, 0, 0, 0, 0, 0),
(26011, 26021, 'author', 'en', '◡#❷❷❷ :: Pokemon hack mod ', 1771, 1, 1468427197, 1468427197, 0, 0, 0, 0, 0, 0),
(26012, 26022, 'author', 'en', '♊[CHEAT] 13.07.201*6 :: Pokemon cheatS Android fRee PokeCoins', 1771, 1, 1468427203, 1468427203, 0, 0, 0, 0, 0, 0),
(26013, 26023, 'author', 'en', '✶{!{haCk}!}✷ 13.07.2016 :: Pokemon ipa hack hack PokeCoins | Andr,oid/iO♷S', 1771, 1, 1468427206, 1468427206, 0, 0, 0, 0, 0, 0),
(26014, 26024, 'author', 'en', '☪❾❾❾☫ :: Pokemon Hack hack PokeBalls | 2016', 1771, 1, 1468427208, 1468427208, 0, 0, 0, 0, 0, 0),
(26015, 26025, 'author', 'en', '◵(*(MEGAHACK)*) :: Pokemon hack tool download PokeCoins, P☓okeBalls | iOS-Android', 1771, 1, 1468427212, 1468427212, 0, 0, 0, 0, 0, 0),
(26016, 26026, 'author', 'en', '◆july◇ 13:07:2016 :: Pokemon hack jaibreak Hack PokeCoins | 2016', 1771, 1, 1468427218, 1468427218, 0, 0, 0, 0, 0, 0),
(26017, 26027, 'author', 'en', '♼(~❹❹❹~)♽ :: Pokemon Triche PokeCoins', 1771, 1, 1468427222, 1468427222, 0, 0, 0, 0, 0, 0),
(26018, 26028, 'author', 'en', '◾{!{❹❹�♗�}!} :: how to hack/ Pokemon hack PokeBalls | iOS-Android', 1771, 1, 1468427226, 1468427226, 0, 0, 0, 0, 0, 0),
(26019, 26029, 'author', 'en', '♈[~❹❹❹~] :: Pokemon cheat sheet free PokeBalls | Android', 1771, 1, 1468427232, 1468427232, 0, 0, 0, 0, 0, 0),
(26020, 26030, 'author', 'en', '▣(~❼❼❼~) :: Pokemon tips for beginners free| PokeBalls | 2016', 1771, 1, 1468427238, 1468427238, 0, 0, 0, 0, 0, 0),
(26021, 26031, 'author', 'en', '☋(!(❽❽❽)!)☋ :: Pokemon Juw.elen Hack free◔ PokeCoins | 2016', 1771, 1, 1468427243, 1468427243, 0, 0, 0, 0, 0, 0),
(26022, 26032, 'author', 'en', '✽~!~❽❽❽✽ :: Pokemon hack real ', 1771, 1, 1468427247, 1468427247, 0, 0, 0, 0, 0, 0),
(26023, 26033, 'author', 'en', '♨{~{❶❶❶✭}~} :: Pokemon cheats for iphone ', 1771, 1, 1468427251, 1468427251, 0, 0, 0, 0, 0, 0),
(26024, 26034, 'author', 'en', '☱(!❶❶❶!)☱ :: Pokemon tips advanced PokeBalls', 1771, 1, 1468427255, 1468427255, 0, 0, 0, 0, 0, 0),
(26025, 26035, 'author', 'en', '❔$c✽heaT❔ :: how to get hack◚ tool Pokemon generate PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427266, 1468427266, 0, 0, 0, 0, 0, 0),
(26026, 26036, 'author', 'en', '☬{!❹❹❹!}☭ :: Pokemon hack android ', 1771, 1, 1468427268, 1468427268, 0, 0, 0, 0, 0, 0),
(26027, 26037, 'author', 'en', '☝(!(CHEAT)!)☞ 13:0♔7:▨2016; :: Pokemon cheats and hacks ', 1771, 1, 1468427274, 1468427274, 0, 0, 0, 0, 0, 0),
(26028, 26038, 'author', 'en', '☐[![hackpa;ck]!] 13:07:2016 :/: Pokemon cheats codes ', 1771, 1, 1468427278, 1468427278, 0, 0, 0, 0, 0, 0),
(26029, 26039, 'author', 'en', '❟{{❹,❹❹☴}}❟ :: Pokemon Hack Cheat Juwe.len Deutsch! generate PokeCoins | 2016', 1771, 1, 1468427283, 1468427283, 0, 0, 0, 0, 0, 0),
(26030, 26040, 'author', 'en', '♟|_/¯❾�.��❾¯\\_| :: No survey hack google play Pokemon  PokeBalls | 2016', 1771, 1, 1468427290, 1468427290, 0, 0, 0, 0, 0, 0),
(26031, 26041, 'author', 'en', '◼{!{❺♝❺❺}!} :: Pokemon Cheats ', 1771, 1, 1468427290, 1468427290, 0, 0, 0, 0, 0, 0),
(26032, 26042, 'author', 'en', '♬!❸❸❸ :: Pokemon hack no j▼aib;rea;k generat●e|❐ PokeCoins, POkeBalls | iOS-Android', 1771, 1, 1468427305, 1468427305, 0, 0, 0, 0, 0, 0),
(26033, 26043, 'author', 'en', '✩{*{❶❶❶}*} :: Pokemon hack online ', 1771, 1, 1468427305, 1468427305, 0, 0, 0, 0, 0, 0),
(26034, 26044, 'author', 'en', '✸{!HACKENGINE!} 13.07.2016 :::: Pokemon hack ge:nerator ', 1771, 1, 1468427321, 1468427321, 0, 0, 0, 0, 0, 0),
(26035, 26045, 'author', 'en', '☡(!HACK!)☢ 13.07.2016 :: Pokemon che^ats onl♶ine ', 1771, 1, 1468427330, 1468427330, 0, 0, 0, 0, 0, 0),
(26036, 26046, 'author', 'en', '☸(*❻❻❻*)☹ :: Pokemon cheat review PokeCoins, PokeBalls | Android', 1771, 1, 1468427332, 1468427332, 0, 0, 0, 0, 0, 0),
(26037, 26047, 'author', 'en', '♯###❻❻❻ :: Pokemon cheat code entry PokeCoIns, PokeBalls | iOS-Andro☐id', 1771, 1, 1468427334, 1468427334, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(26038, 26048, 'author', 'en', '☕###2016☕ :: Pokemon tips tricks PokeBalls | 2016', 1771, 1, 1468427338, 1468427338, 0, 0, 0, 0, 0, 0),
(26039, 26049, 'author', 'en', '▧[![❽❽�♥�]!] :: Pokemon pirater teLecharg,er PokeCoins', 1771, 1, 1468427343, 1468427343, 0, 0, 0, 0, 0, 0),
(26040, 26050, 'author', 'en', '○{!{❼❼❼}!}◌ :: Pokemon cheats and haCks◐ hack PokeBalls | iOS-Android', 1771, 1, 1468427349, 1468427349, 0, 0, 0, 0, 0, 0),
(26041, 26051, 'author', 'en', '◞{❶❶❶}◟ :: Pokemon tips advanced ', 1771, 1, 1468427353, 1468427353, 0, 0, 0, 0, 0, 0),
(26042, 26052, 'author', 'en', '▨(*❼❼❼*)▨ :: Pokemon Pirater PokeCOins, PokeBalls | And.roid/iOS', 1771, 1, 1468427354, 1468427354, 0, 0, 0, 0, 0, 0),
(26043, 26053, 'author', 'en', '☼###GENERATOR :: Pokemon hack android no roO◭t ', 1771, 1, 1468427358, 1468427358, 0, 0, 0, 0, 0, 0),
(26044, 26054, 'author', 'en', '▫_/¯❾❾❾¯\\_▬ :: Pokemon cheats codes generate PokeBalls', 1771, 1, 1468427363, 1468427363, 0, 0, 0, 0, 0, 0),
(26045, 26055, 'author', 'en', '♨(~(07-13-2016)~)♩ :: no survey hack google play◔ Pokemon  free P◟okeCoins, PokeBalls | iOS-Android', 1771, 1, 1468427367, 1468427367, 0, 0, 0, 0, 0, 0),
(26046, 26056, 'author', 'en', '✡(!HACKPACK!) 13♄:07:2016 ::✠ Pokemon hack tool download PokeBalls | 2016', 1771, 1, 1468427373, 1468427373, 0, 0, 0, 0, 0, 0),
(26047, 26057, 'author', 'en', '▰{{2016}} 13/07/20♒16 :: how to hack Pokemon ', 1771, 1, 1468427377, 1468427377, 0, 0, 0, 0, 0, 0),
(26048, 26058, 'author', 'en', '✹[*[❷❷❷]*] :: Astuces Pour Pokemon free PokeCoinS | Android', 1771, 1, 1468427381, 1468427381, 0, 0, 0, 0, 0, 0),
(26049, 26059, 'author', 'en', '✰((❽❽❽))✱ :: Pokemon Triche ', 1771, 1, 1468427394, 1468427394, 0, 0, 0, 0, 0, 0),
(26050, 26060, 'author', 'en', '◓[~[❼❼❼]~] :: Pokemon hack download .', 1771, 1, 1468427397, 1468427397, 0, 0, 0, 0, 0, 0),
(26051, 26061, 'author', 'en', '♓❽❽❽♓ :: Pokemon hack iphone free PokeCoins, PokeBalls | 2016', 1771, 1, 1468427399, 1468427399, 0, 0, 0, 0, 0, 0),
(26052, 26062, 'author', 'en', '■(*❹❹❹*)■ :: Pokemon hack android hack POkeBa◐lls❐', 1771, 1, 1468427404, 1468427404, 0, 0, 0, 0, 0, 0),
(26053, 26063, 'author', 'en', '✓[[engi..n♸e]]✓ 13.07.2016 :.: Pokemon Hack Deu.tsch PokeBalls', 1771, 1, 1468427410, 1468427410, 0, 0, 0, 0, 0, 0),
(26054, 26064, 'author', 'en', '™[*gEnera.tor*] 13-07-2016 :: Pokemon hack genera❖toR ', 1771, 1, 1468427430, 1468427430, 0, 0, 0, 0, 0, 0),
(26055, 26065, 'author', 'en', '◯[~[fullhack]~] 13-07-2016 :: Pokemon Hack Cheats Trainer ', 1771, 1, 1468427430, 1468427430, 0, 0, 0, 0, 0, 0),
(26056, 26066, 'author', 'en', '◾[~❹❹❹~]◿ :: Pokemon Triche Gratuit PokeBalls | iOS-A:ndroid', 1771, 1, 1468427431, 1468427431, 0, 0, 0, 0, 0, 0),
(26057, 26067, 'author', 'en', '▸❶❶❶▹ :: Pokemon Mod Apk ', 1771, 1, 1468427432, 1468427432, 0, 0, 0, 0, 0, 0),
(26058, 26068, 'author', 'en', '❛|_/¯�.�❻❻¯\\_|❛ :: Pokemon Hack Tool APK IPA ', 1771, 1, 1468427453, 1468427453, 0, 0, 0, 0, 0, 0),
(26059, 26069, 'author', 'en', '◐{!{hackcheats}!} :: Pokemon Glitch and Glitches ', 1771, 1, 1468427460, 1468427460, 0, 0, 0, 0, 0, 0),
(26060, 26070, 'author', 'en', '▼$❺❺❺▼ :: Pokemon hack fOr ios free PokeCoins | Android', 1771, 1, 1468427460, 1468427460, 0, 0, 0, 0, 0, 0),
(26061, 26071, 'author', 'en', '✂(*�.��*❸❸*) :: Pokemon cheat sheet PokeCoins, PokeBaLls | iOS-Android', 1771, 1, 1468427475, 1468427475, 0, 0, 0, 0, 0, 0),
(26062, 26072, 'author', 'en', '♎{!{❼❼❼}!} :: Pokemon hack ifunbox hack Pok◀eCoins | Android/iOS', 1771, 1, 1468427481, 1468427481, 0, 0, 0, 0, 0, 0),
(26063, 26073, 'author', 'en', '◌[~GETHACK~] 13-07♴-2016 :: Pokemon hack work P/o/k/e/C/o/i/n/s/ | 2016', 1771, 1, 1468427481, 1468427481, 0, 0, 0, 0, 0, 0),
(26064, 26074, 'author', 'en', '◞[![GENERATOR]!] 13.07..2/016 :: Pokemon Mod A◿pk hack PokeBalls', 1771, 1, 1468427484, 1468427484, 0, 0, 0, 0, 0, 0),
(26065, 26075, 'author', 'en', '✫#❹❹❹✬ :: Pokemon cheats hacks free Poke◁Coins', 1771, 1, 1468427486, 1468427486, 0, 0, 0, 0, 0, 0),
(26066, 26076, 'author', 'en', '☾[*HACKCHEATS*] 13.07.2016 :: Pokemon Hacker PokeCoins', 1771, 1, 1468427496, 1468427496, 0, 0, 0, 0, 0, 0),
(26067, 26077, 'author', 'en', '☣[!❺❺❺!] :: Pokemon cheats real PokeBalls | Android/iOS', 1771, 1, 1468427502, 1468427502, 0, 0, 0, 0, 0, 0),
(26068, 26078, 'author', 'en', '☬(!o\\nline!)☭ 13./07.2016 :: Pokemon hack ios h.a.c.k. PokeBalls | Andr/oid/iOS', 1771, 1, 1468427505, 1468427505, 0, 0, 0, 0, 0, 0),
(26069, 26079, 'author', 'en', '™(*❺❺❺*)© :: Pokemon Hack Deutsch ', 1771, 1, 1468427506, 1468427506, 0, 0, 0, 0, 0, 0),
(26070, 26080, 'author', 'en', '♗{*MEGAHACK*}♘ 13-07-2016 :: Pokemon h:a:c:k: that works ^', 1771, 1, 1468427508, 1468427508, 0, 0, 0, 0, 0, 0),
(26071, 26081, 'author', 'en', '☫{!❶❶❶!}☫ :: Pokemon Juwelen Cheat hack Po|keC|oins, PokeBall.◮s', 1771, 1, 1468427516, 1468427516, 0, 0, 0, 0, 0, 0),
(26072, 26082, 'author', 'en', '✸###HACKTOOL✹ :: how to get. haCk tool Pokemon ', 1771, 1, 1468427523, 1468427523, 0, 0, 0, 0, 0, 0),
(26073, 26083, 'author', 'en', '♋(*2016*)♋ 13-07-2016 :: Pokemon cheat tool ', 1771, 1, 1468427527, 1468427527, 0, 0, 0, 0, 0, 0),
(26074, 26084, 'author', 'en', '♄(~(�◢�❾❾)~)♅ ::♬ Pokemon cheats f.or ipad PokeCoins', 1771, 1, 1468427540, 1468427540, 0, 0, 0, 0, 0, 0),
(26075, 26085, 'author', 'en', '♧|¯\\_CHEATS_/¯|♨ 13.07.2016 :: Pokemon hack safe gene.ra✱te PokeCoins | iOS-Android', 1771, 1, 1468427548, 1468427548, 0, 0, 0, 0, 0, 0),
(26076, 26086, 'author', 'en', '◛[![❹❹❹]!]◛ :: Pokemon hack mod PokeCoins | 2016', 1771, 1, 1468427551, 1468427551, 0, 0, 0, 0, 0, 0),
(26077, 26087, 'author', 'en', '□#�.�.�.�.�.�.�.�.�.□ :: Pokemon hack ifile PokeBalls | Android', 1771, 1, 1468427551, 1468427551, 0, 0, 0, 0, 0, 0),
(26078, 26088, 'author', 'en', '♨¯\\_engiNe_/¯ 13-07-2016 :: Pokemon tips and cheats ', 1771, 1, 1468427562, 1468427562, 0, 0, 0, 0, 0, 0),
(26079, 26089, 'author', 'en', '◃[*[❾❾❾]*]◃ :: Pokemon Triche Gratuit Po◨keCoins, PokeBa♫lls☈ | Android/iOS', 1771, 1, 1468427568, 1468427568, 0, 0, 0, 0, 0, 0),
(26080, 26090, 'author', 'en', '♀|¯\\_❾❾❾.._/¯|♀ :: Pokemon Hack Cheat Juwele.n Deutsch! hack PokeCoins | iOS-Android', 1771, 1, 1468427572, 1468427572, 0, 0, 0, 0, 0, 0),
(26081, 26091, 'author', 'en', '▻[!❸�:�❸!] :*:*  PokeCoins, PokEBalls | iOS-Android', 1771, 1, 1468427573, 1468427573, 0, 0, 0, 0, 0, 0),
(26082, 26092, 'author', 'en', '☉(!(ENGINE)!) 13.07.2016 :: Pokemon cheat She◐et ', 1771, 1, 1468427586, 1468427586, 0, 0, 0, 0, 0, 0),
(26083, 26093, 'author', 'en', '☇[HACKENGINE]☈ 13-07-2016 :: Pokemon hack and cheats free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468427587, 1468427587, 0, 0, 0, 0, 0, 0),
(26084, 26094, 'author', 'en', '▼{!07-13-2016!}▽ :: Pokemon Pirater .', 1771, 1, 1468427592, 1468427592, 0, 0, 0, 0, 0, 0),
(26085, 26095, 'author', 'en', '☦[07-13-2016] :: Pokemon Hack Cheats Trainer PokeBalls | iO|S-Android', 1771, 1, 1468427608, 1468427608, 0, 0, 0, 0, 0, 0),
(26086, 26096, 'author', 'en', '☶_/¯❻�.�❻¯\\_☶ :: Pokemon hack downl►oad ', 1771, 1, 1468427612, 1468427612, 0, 0, 0, 0, 0, 0),
(26087, 26097, 'author', 'en', '♮(!❾❾❾!) :: Pokemon Glitch ', 1771, 1, 1468427614, 1468427614, 0, 0, 0, 0, 0, 0),
(26088, 26098, 'author', 'en', '✂generat✣or✒ 13-✶07-2016 :: Pokemon hack ifunbox g.e.n.e.r.a.t.e. P\\okeCoins', 1771, 1, 1468427623, 1468427623, 0, 0, 0, 0, 0, 0),
(26089, 26099, 'author', 'en', '☚|_/¯❽❽❽¯\\_|☛ :: Pokemon cheats hacks ', 1771, 1, 1468427624, 1468427624, 0, 0, 0, 0, 0, 0),
(26090, 26100, 'author', 'en', '[@ SKY!! TV @]Basel vs Zenit St. Petersburg Live Stream ', 2057, 1, 1468427647, 1468427725, 0, 0, 0, 0, 0, 0),
(26091, 26101, 'author', 'en', '☃(!❾❾,❾!) :: Pokemon cheats free ', 1771, 1, 1468427652, 1468427652, 0, 0, 0, 0, 0, 0),
(26092, 26102, 'author', 'en', '☭[~❶❶❶~]☮ :: Pokemon cheat code entry PokeCoins | Android', 1771, 1, 1468427654, 1468427654, 0, 0, 0, 0, 0, 0),
(26093, 26103, 'author', 'en', '☕{!{❶❶❶}!}☕ :: Pokemon p▾ir❏ater telecharger ', 1771, 1, 1468427655, 1468427655, 0, 0, 0, 0, 0, 0),
(26094, 26104, 'author', 'en', '❇❾❾❾ :: Pokemon ipa hack ', 1771, 1, 1468427657, 1468427657, 0, 0, 0, 0, 0, 0),
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(26096, 26106, 'author', 'en', '⚓{*{❻❻❻}*}﨏 :: Pokemon cheats tool ', 1771, 1, 1468427675, 1468427675, 0, 0, 0, 0, 0, 0),
(26097, 26107, 'author', 'en', '◉(!(GEN☲ERATOR)!)◉ 13.07.2016 :: Pokemon cheats reviews generate P.okeBalls | And*ro*id', 1771, 1, 1468427678, 1468427678, 0, 0, 0, 0, 0, 0),
(26098, 26108, 'author', 'en', '◓(!hackgene,rator!) 1.3./.0.7./.2.0.1.6. :: Pokemon Hack Gratis PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468427681, 1468427681, 0, 0, 0, 0, 0, 0),
(26099, 26109, 'author', 'en', '◃{CHEAT}◃ 13.07.2016 :: Pokemon hack that works free PokeCoins | 2016', 1771, 1, 1468427683, 1468427683, 0, 0, 0, 0, 0, 0),
(26100, 26110, 'author', 'en', '☦#CHEATS☧ 13.07.2016 :: Pokemon cheat shee|t ha\\ck PoKeCoins | A♃ndroid/iOS', 1771, 1, 1468427699, 1468427699, 0, 0, 0, 0, 0, 0),
(26101, 26111, 'author', 'en', '♪{~{july}~} 13/07/2016 :: Pokemon Astuce gEnerAte PokeBalls | iOS-Android', 1771, 1, 1468427701, 1468427701, 0, 0, 0, 0, 0, 0),
(26102, 26112, 'author', 'en', '◆mod◆ 13/07/2016 :: Pokemon Online Hack ', 1771, 1, 1468427706, 1468427706, 0, 0, 0, 0, 0, 0),
(26103, 26113, 'author', 'en', '◶{!❹❹❹!}◷ :: Pokemon chea♏ts hacks hack PoKeCoins | AnDroid/iOS', 1771, 1, 1468427706, 1468427706, 0, 0, 0, 0, 0, 0),
(26104, 26114, 'author', 'en', '✖{!{❼.�..�^�❼}!} :: Pokemon tips tRicks ', 1771, 1, 1468427717, 1468427717, 0, 0, 0, 0, 0, 0),
(26105, 26115, 'author', 'en', '☖[*❺❺❺*]☗ :: Pokemon Cheats 2016 generate PokeCoins | 2016', 1771, 1, 1468427720, 1468427720, 0, 0, 0, 0, 0, 0),
(26106, 26116, 'author', 'en', '✼{❻❻❻} :: Pokemon cheat real hack PokeCoins, PokeBalls', 1771, 1, 1468427723, 1468427723, 0, 0, 0, 0, 0, 0),
(26107, 26117, 'author', 'en', '♂[*[❺❺❺]*] :: Pokemon pirater telechargeR PokeBalls | Android/iOS', 1771, 1, 1468427728, 1468427728, 0, 0, 0, 0, 0, 0),
(26108, 26118, 'author', 'en', '✺{~ENGINE~}✻ 13.07.2016 :: Pokemon cheat review ', 1771, 1, 1468427738, 1468427738, 0, 0, 0, 0, 0, 0),
(26109, 26119, 'author', 'en', '♾❶❶❶:♿ :: Pokemon Glitch and Glitches ', 1771, 1, 1468427752, 1468427752, 0, 0, 0, 0, 0, 0),
(26110, 26120, 'author', 'en', '✩{*{hacked}*}✩ :: Pokemon HAck 2016 PokeCoins, PokeBalls', 1771, 1, 1468427762, 1468427762, 0, 0, 0, 0, 0, 0),
(26111, 26121, 'author', 'en', '♕{!july!}♖ 13-07-2016 :: Pokemon Hack Cheat Juwelen DEutsch! ', 1771, 1, 1468427764, 1468427764, 0, 0, 0, 0, 0, 0),
(26112, 26122, 'author', 'en', '☱[*[hackengine]*]☱ 13-07-2016 :: Pokemon tips for beginners gene✲rate POkeBAll☂s | iOS-AndroID', 1771, 1, 1468427772, 1468427772, 0, 0, 0, 0, 0, 0),
(26113, 26123, 'author', 'en', '▶(!(HACKCHEATS)!) :|:| Pokemon hack android no root Hack POkeCoins | Android/iOS', 1771, 1, 1468427773, 1468427773, 0, 0, 0, 0, 0, 0),
(26114, 26124, 'author', 'en', '☣(~❻❻❻~)☣ :: Pokemon cheats and hacks ', 1771, 1, 1468427776, 1468427776, 0, 0, 0, 0, 0, 0),
(26115, 26125, 'author', 'en', '✗{~hacked~}✗ 13/07/2016 ::☆ Pokemon hack generator ☥', 1771, 1, 1468427790, 1468427790, 0, 0, 0, 0, 0, 0),
(26116, 26126, 'author', 'en', '♀[!❽❽❽!]♁ :|:| Pokemon Hack no jaibrea◛k generate Po:keBalls | Android', 1771, 1, 1468427791, 1468427791, 0, 0, 0, 0, 0, 0),
(26117, 26127, 'author', 'en', '◮[!❽❽❽!]◮ :: Pokemon Astuce ', 1771, 1, 1468427800, 1468427800, 0, 0, 0, 0, 0, 0),
(26118, 26128, 'author', 'en', '♖❺..❺❺♖ :: Pokemon hACk generator ', 1771, 1, 1468427800, 1468427800, 0, 0, 0, 0, 0, 0),
(26119, 26129, 'author', 'en', '❍{!onlin♧e!} 13-☍07-20.16 :: Hack Para Pokemon hack POkeCoins, PokeBalls | iOS-Android', 1771, 1, 1468427804, 1468427804, 0, 0, 0, 0, 0, 0),
(26120, 26130, 'author', 'en', '◚[!❺❺❺!]◚ :: Pokemon hack a.p.k. ', 1771, 1, 1468427817, 1468427817, 0, 0, 0, 0, 0, 0),
(26121, 26131, 'author', 'en', '▩{!{❽❽❽}!}▪ :: Pokemon cheat th|at works PokECoins❛, PokeBalls | A/n/d/r/o/i/d/', 1771, 1, 1468427818, 1468427818, 0, 0, 0, 0, 0, 0),
(26122, 26132, 'author', 'en', '❇{*❺�.�❺*}❇ :*:* Pokemon Juwelen HacK generate PokeBalLs | A^n^d^r^o^i^d^', 1771, 1, 1468427821, 1468427821, 0, 0, 0, 0, 0, 0),
(26123, 26133, 'author', 'en', '♫❻❻❻♬ :: Pokemon Tips and Tricks ', 1771, 1, 1468427821, 1468427821, 0, 0, 0, 0, 0, 0),
(26124, 26134, 'author', 'en', '□~!~❾❾�♎��▢ :: Pokemon Astuce PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427838, 1468427838, 0, 0, 0, 0, 0, 0),
(26125, 26135, 'author', 'en', '♸_/¯�♞��❶❶¯\\_♸ ::☦ Pokemon Hack iPhone i♹Pad Android PokeBalls | Android/iOS', 1771, 1, 1468427842, 1468427842, 0, 0, 0, 0, 0, 0),
(26126, 26136, 'author', 'en', '~!~❷❷.❷ :: Pokemon hack ipad free PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427845, 1468427845, 0, 0, 0, 0, 0, 0),
(26127, 26137, 'author', 'en', '❔{{❶❶�◻�}}❔ :: Pokemon cheats iphone free PokeBalls | Android/iOS', 1771, 1, 1468427860, 1468427860, 0, 0, 0, 0, 0, 0),
(26128, 26138, 'author', 'en', '▽{!mod!}▽ 13-07-2016 :: Pokemon hack online generate PokeCoins, PokeBalls | iOS-AndrOid', 1771, 1, 1468427862, 1468427862, 0, 0, 0, 0, 0, 0),
(26129, 26139, 'author', 'en', '▬(!(O!NLINE)!)▭ :: Pokemon cheat codes ', 1771, 1, 1468427866, 1468427866, 0, 0, 0, 0, 0, 0),
(26130, 26140, 'author', 'en', '◳#❸❸❸ :: Pokemon Hack 2016 PokeCoins, PokeBalls | 2016', 1771, 1, 1468427867, 1468427867, 0, 0, 0, 0, 0, 0),
(26131, 26141, 'author', 'en', '☰[!hackcheats!]☱ 13:07:2016 :: Pokemon Triche f/r/e/e/ P,okeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427871, 1468427871, 0, 0, 0, 0, 0, 0),
(26132, 26142, 'author', 'en', '♔[*[❺❺❺]*]♔ :◭: Pokemon hack jAibreak ', 1771, 1, 1468427884, 1468427884, 0, 0, 0, 0, 0, 0),
(26133, 26143, 'author', 'en', '▸~!~Cheats◕▸ 13:07:2016 :: Pokemon cheat engine free PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468427891, 1468427891, 0, 0, 0, 0, 0, 0),
(26134, 26144, 'author', 'en', '◛{*❸❸❸*}◛ :: Pokemon c,h,e,a,t,s, iphone ', 1771, 1, 1468427907, 1468427907, 0, 0, 0, 0, 0, 0),
(26135, 26145, 'author', 'en', '♎(*HACKTOOL*) 13/07/2016 :: Pokemon cheats iphone ', 1771, 1, 1468427908, 1468427908, 0, 0, 0, 0, 0, 0),
(26136, 26146, 'author', 'en', '☪{~hAcked~} 1.3/07/2016 :: Pokemon Cheats ', 1771, 1, 1468427910, 1468427910, 0, 0, 0, 0, 0, 0),
(26137, 26147, 'author', 'en', '♊(~❸❸❸~)♊ :☦: Pokemon haCk ifunbox generate P►okeBalls | Android', 1771, 1, 1468427911, 1468427911, 0, 0, 0, 0, 0, 0),
(26138, 26148, 'author', 'en', '♘{!{hacktool}!} 13/07/2016 :: .. free P☈o^keBalls | Android', 1771, 1, 1468427916, 1468427916, 0, 0, 0, 0, 0, 0),
(26139, 26149, 'author', 'en', '◡hackpAck◢ :: Pokemon cheats ios hack PokeCoins, PokeBalls | 2016', 1771, 1, 1468427931, 1468427931, 0, 0, 0, 0, 0, 0),
(26140, 26150, 'author', 'en', '♘{megahack} 13-07-2016 :: Pokemon hack download geneRate PokeCoins | Android/iOS', 1771, 1, 1468427935, 1468427935, 0, 0, 0, 0, 0, 0),
(26141, 26151, 'author', 'en', '♄((mod)) 13..07.2016 :: Pokemon Generator free PokeBalls | iOS-Android', 1771, 1, 1468427937, 1468427937, 0, 0, 0, 0, 0, 0),
(26142, 26152, 'author', 'en', '♎[![❾❾�★��]!]♎ :: Astuces Pour Pokemon ', 1771, 1, 1468427937, 1468427937, 0, 0, 0, 0, 0, 0),
(26143, 26153, 'author', 'en', '▥{*CHEATS*}▥ 13.07.2016 :: Pokemon hac◘k i|phone ', 1771, 1, 1468427938, 1468427938, 0, 0, 0, 0, 0, 0),
(26144, 26154, 'author', 'en', '◪|_/¯fullhack¯\\_|◪ 13-07-2.016 :: Pokemon ti.ps and tricks PokeB◎alls | iOS-Android', 1771, 1, 1468427956, 1468427956, 0, 0, 0, 0, 0, 0),
(26145, 26155, 'author', 'en', '▩HAC◗KGENERATOR 13:07:2016 :: Pokemon cheats free hack♣ PokeCoins | 2016', 1771, 1, 1468427961, 1468427961, 0, 0, 0, 0, 0, 0),
(26146, 26156, 'author', 'en', '☣(cheats) 13-07-2016 :: Pokemon cheat☰ code entry generate PokeCoins | 2016', 1771, 1, 1468427964, 1468427964, 0, 0, 0, 0, 0, 0),
(26147, 26157, 'author', 'en', '♱(~❶❶❶~)♲ :: Pokemon hack online generate PokeCoins | Andro◢id', 1771, 1, 1468427965, 1468427965, 0, 0, 0, 0, 0, 0),
(26148, 26158, 'author', 'en', '♴[*[generator]*]♴ 13.07.2016 :: Pokemon Hacken hac❑k PokeBalLS | Android/iOS', 1771, 1, 1468427966, 1468427966, 0, 0, 0, 0, 0, 0),
(26149, 26159, 'author', 'en', '▲[~[❻❻❻]~]▲ :: Pokemon hack tool download ', 1771, 1, 1468427982, 1468427982, 0, 0, 0, 0, 0, 0),
(26150, 26160, 'author', 'en', '◙!hackengine◚ 13.07.2016 :: Pokemon hack for android hac☲k PokeCoins | iOS-Androi..d', 1771, 1, 1468427983, 1468427983, 0, 0, 0, 0, 0, 0),
(26151, 26161, 'author', 'en', '♾¯\\_FULLHACK_/¯ :: Pokemon cheats for i◨phone generate PokeBalls | 2016', 1771, 1, 1468427989, 1468427989, 0, 0, 0, 0, 0, 0),
(26152, 26162, 'author', 'en', '□(hackgen.erator)▢ 13-07-2016 :: Pokemon Online Ha..ck PokeCoins | Android', 1771, 1, 1468428006, 1468428006, 0, 0, 0, 0, 0, 0),
(26153, 26163, 'author', 'en', '✽(!�*�❸❸!)✾ :: Pokemon Hack PokeCoins', 1771, 1, 1468428007, 1468428007, 0, 0, 0, 0, 0, 0),
(26154, 26164, 'author', 'en', '♗(!(❺❺❺)!)♘ :: Pokemon Astuce free PokeBalls | Android/iOS', 1771, 1, 1468428014, 1468428014, 0, 0, 0, 0, 0, 0),
(26155, 26165, 'author', 'en', '◮###❹❹▨❹ ::^ Pokemon ch*eat code entry PokeBall!s |❖ 20◸16', 1771, 1, 1468428014, 1468428014, 0, 0, 0, 0, 0, 0),
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(26157, 26167, 'author', 'en', '◗|¯\\_�;��❹❹_/¯| :: no survey hack google pLay Pokemon  hack Pok.eCoins | 2016', 1771, 1, 1468428039, 1468428039, 0, 0, 0, 0, 0, 0),
(26158, 26168, 'author', 'en', '✮❾❾❾ :: Pokemon cheats for iphone free PokeCoins | iOS-Androi|d', 1771, 1, 1468428040, 1468428040, 0, 0, 0, 0, 0, 0),
(26159, 26169, 'author', 'en', '◯[![CHEAT]!] 13-07-2016 :: Pokemon hack tool dOw,nload ', 1771, 1, 1468428054, 1468428054, 0, 0, 0, 0, 0, 0),
(26160, 26170, 'author', 'en', '✒[![❷❷❷]!]✩ :: Pokemon ipa hack generate PokeBalls', 1771, 1, 1468428055, 1468428055, 0, 0, 0, 0, 0, 0),
(26161, 26171, 'author', 'en', '▼{❹�◛�❹}▽ :: Pokemon cheAts for mobile hack PokeCoins, PokeBalls | Andr.oid/iOS', 1771, 1, 1468428064, 1468428064, 0, 0, 0, 0, 0, 0),
(26162, 26172, 'author', 'en', '☗((❻❻�✹�))☗ :: Pokemon hack ios ', 1771, 1, 1468428067, 1468428067, 0, 0, 0, 0, 0, 0),
(26163, 26173, 'author', 'en', '◑|_/¯�✯��❶❶¯\\_|◑ :: Pokemon HAck.en PokeCoins | iOS-Android', 1771, 1, 1468428068, 1468428068, 0, 0, 0, 0, 0, 0),
(26164, 26174, 'author', 'en', '☥{!{❸❸❸}!}☥ :: Pokemon Tips and Tricks ', 1771, 1, 1468428081, 1468428081, 0, 0, 0, 0, 0, 0),
(26165, 26175, 'author', 'en', '▨{!{❽❽❽}!}▨ :: Pokemon cheats for ipad generate PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468428084, 1468428084, 0, 0, 0, 0, 0, 0),
(26166, 26176, 'author', 'en', '✭(!(HA❰CKTOOL)!)✭ :: Pokemon Pirater free PokeBalls', 1771, 1, 1468428090, 1468428090, 0, 0, 0, 0, 0, 0),
(26167, 26177, 'author', 'en', '◕~!~HACKPAC✡K 13.07.2016 :: Pokemon cheats hacking hack PokeBalls | iOS-Android', 1771, 1, 1468428093, 1468428093, 0, 0, 0, 0, 0, 0),
(26168, 26178, 'author', 'en', '©[*[❽❽❽]*]© :: Pokemon hack for android f^r^e^e^ PokeCoins.', 1771, 1, 1468428096, 1468428096, 0, 0, 0, 0, 0, 0),
(26169, 26179, 'author', 'en', '♁|_/¯ON,LINE¯\\_|♂ 13-07-2016 :: Pokemon c.heat that works generate PokeCoins, PokeBalls | iOS-Andro^id', 1771, 1, 1468428108, 1468428108, 0, 0, 0, 0, 0, 0),
(26170, 26180, 'author', 'en', '☦((❾❾:❾))☧ :: Pokemon Mod Apk ', 1771, 1, 1468428113, 1468428113, 0, 0, 0, 0, 0, 0),
(26171, 26181, 'author', 'en', '❜_/¯❼❼❼¯\\_❜ :: Pokemon Hacker PokeCoins | AndroId/iOS', 1771, 1, 1468428113, 1468428113, 0, 0, 0, 0, 0, 0),
(26172, 26182, 'author', 'en', '✻F.U.L.L.H.A.C.K.✻ 13:07:/2016 ::.. Pokemon apk cheats PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468428115, 1468428115, 0, 0, 0, 0, 0, 0),
(26173, 26183, 'author', 'en', '❗[![GETHACK]!] :: Pokemon hack mac PokeBalls | Android', 1771, 1, 1468428121, 1468428121, 0, 0, 0, 0, 0, 0),
(26174, 26184, 'author', 'en', '☇{*{ENGINE}*}☈ 13.07.2016 :: Pokemon pirater telecharger ha.ck PokeCoins | iOS-Android', 1771, 1, 1468428131, 1468428131, 0, 0, 0, 0, 0, 0),
(26175, 26185, 'author', 'en', '❕[![ONLINE]!]❕ 13:07:2016 :: Pokemon Triche Grat®uit❏ generate Poke♨Coins | Android', 1771, 1, 1468428136, 1468428136, 0, 0, 0, 0, 0, 0),
(26176, 26186, 'author', 'en', '❞###❽❽❽❞ :: Pokemon Hack Cheat Juwelen Deutsch! ', 1771, 1, 1468428148, 1468428148, 0, 0, 0, 0, 0, 0),
(26177, 26187, 'author', 'en', '▢{*❽❽❽*} :: Pokemon Hack Gratis hack PokeCoins, PokeBalls | AndRoid/iOS', 1771, 1, 1468428157, 1468428157, 0, 0, 0, 0, 0, 0),
(26178, 26188, 'author', 'en', '❁[![❸❸❸]!]❁ :: Pokemon Tips and Tricks hack PokeBalls | iOS-Android', 1771, 1, 1468428161, 1468428161, 0, 0, 0, 0, 0, 0),
(26179, 26189, 'author', 'en', '◜{~❷❷❷~} ::☍ Pokemon h♓ack mod free PokeCoins', 1771, 1, 1468428161, 1468428161, 0, 0, 0, 0, 0, 0),
(26180, 26190, 'author', 'en', '♂[~❺❺❺~]♂ :: Pokemon hack too.l free PokeBalls | 2016', 1771, 1, 1468428171, 1468428171, 0, 0, 0, 0, 0, 0),
(26181, 26191, 'author', 'en', '⚓(*(fullhack)*)﨏 1^3.07.2016 :: Pokemon hack no jailbreak PokeCoins, PokeBalls', 1771, 1, 1468428175, 1468428175, 0, 0, 0, 0, 0, 0),
(26182, 26192, 'author', 'en', '◡[*[cheat]*]◢ 13:0;7:2016 :: Pokemon Cheats 2016 hack PokeBalls | Android', 1771, 1, 1468428185, 1468428185, 0, 0, 0, 0, 0, 0),
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(26185, 26195, 'author', 'en', '▶(~hackengine~)▶ 13/07/2016 :: Pokemon T.ips a♓nd Tricks hack P▬okeCoins | Android/iOS', 1771, 1, 1468428208, 1468428208, 0, 0, 0, 0, 0, 0),
(26186, 26196, 'author', 'en', '☔[![❻�..�❻]!] :: Pokemon Hack CheatS Trainer hack PokeCoins', 1771, 1, 1468428210, 1468428210, 0, 0, 0, 0, 0, 0),
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(26188, 26198, 'author', 'en', '◆(!H❆ACKPACK!)◇ :: Code de Triche Pokemon hack PokeBalls | iOS-Android', 1771, 1, 1468428221, 1468428221, 0, 0, 0, 0, 0, 0),
(26189, 26199, 'author', 'en', '♳(~❶❶❶~)♳ :: Pokemon hack free generate Poke.Balls | iOS-A.ndroid', 1771, 1, 1468428229, 1468428229, 0, 0, 0, 0, 0, 0),
(26190, 26200, 'author', 'en', '✤(!(HACKTOOL)!) 13/07/2016 :: Pokemon cheats for ipad generate PokeBalls |! iOS-Android', 1771, 1, 1468428231, 1468428231, 0, 0, 0, 0, 0, 0),
(26191, 26201, 'author', 'en', '✉~!~online✉ 13:07:2016 :: Pokemon hack no survey,  ', 1771, 1, 1468428233, 1468428233, 0, 0, 0, 0, 0, 0),
(26192, 26202, 'author', 'en', '✽[❺�;��❺]✽ :: Pokemon cheAts that work PokeCoins | iOS-Android', 1771, 1, 1468428236, 1468428236, 0, 0, 0, 0, 0, 0),
(26193, 26203, 'author', 'en', '◊(!❽�!��❽!)◊ :: Pokemon cheat download hack PokeBalLs | Android/iOS', 1771, 1, 1468428247, 1468428247, 0, 0, 0, 0, 0, 0),
(26194, 26204, 'author', 'en', '☳[![�♪��❽❽]!]☳ :: Pokemon hack ios PokeCoins | Android/iOS', 1771, 1, 1468428256, 1468428256, 0, 0, 0, 0, 0, 0),
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(26196, 26206, 'author', 'en', '❃[[❶❶❶]] :: Pokemon c◗heats for ipad, PokeCoins | Android/iOS', 1771, 1, 1468428261, 1468428261, 0, 0, 0, 0, 0, 0),
(26197, 26207, 'author', 'en', '◤#❻❻�◅��◥ :: Pokemon hack no root free PokeCoins | iOS-Android', 1771, 1, 1468428263, 1468428263, 0, 0, 0, 0, 0, 0),
(26198, 26208, 'author', 'en', 'Football+>>St Etienne vs PSV l.i.v.e .s.t.r.e.a.m', 2057, 1, 1468428272, 1468428272, 0, 0, 0, 0, 0, 0),
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(26200, 26210, 'author', 'en', '◐|¯\\_07.13.2016_/¯|◑ :: Pokemon hack tool generator hack P*okeCoin^s, PokeBalLs', 1771, 1, 1468428284, 1468428284, 0, 0, 0, 0, 0, 0),
(26201, 26211, 'author', 'en', '✖❾❾..❾❰ :: Pokemon hack safe hack PokeCoins, PokeBalls | 2016', 1771, 1, 1468428288, 1468428288, 0, 0, 0, 0, 0, 0),
(26202, 26212, 'author', 'en', '◓{!{HACKED}!}◓ 13/07/2016 :: how to hack Pokemon ', 1771, 1, 1468428291, 1468428291, 0, 0, 0, 0, 0, 0),
(26203, 26213, 'author', 'en', '✡{~❻❻❻~} :: Pokemon cheat sheet PokeBa✖lls | Android', 1771, 1, 1468428307, 1468428307, 0, 0, 0, 0, 0, 0),
(26204, 26214, 'author', 'en', '◃~!~❶❶❶◃ :: Pokemon Hacker generate PokeCoins | 2016', 1771, 1, 1468428310, 1468428310, 0, 0, 0, 0, 0, 0),
(26205, 26215, 'author', 'en', '✯(*fullhack*)✯ 1,3:07:2016 :: Pokemon Triche Gratuit generate PokeBalls | 2016', 1771, 1, 1468428315, 1468428315, 0, 0, 0, 0, 0, 0),
(26206, 26216, 'author', 'en', '☿[~❸❸❸~] :: Pokemon hack for android hack PokeCoins | 201♡6', 1771, 1, 1468428316, 1468428316, 0, 0, 0, 0, 0, 0),
(26207, 26217, 'author', 'en', '✈(!�◭��❼❼!) :: Pokemon cheats coDes PokeCoins, PokeBalls | iOS-;And.roid', 1771, 1, 1468428327, 1468428327, 0, 0, 0, 0, 0, 0),
(26208, 26218, 'author', 'en', '◐(!hacked!) 13/07^/2016 :: h❋ow to hack Pokemon hack PokeCoins, PokeBalls | Android', 1771, 1, 1468428339, 1468428339, 0, 0, 0, 0, 0, 0),
(26209, 26219, 'author', 'en', '☣(!❽❽❽!)☣ :: Pokemon hack mod apk PokeBalls | Android', 1771, 1, 1468428342, 1468428342, 0, 0, 0, 0, 0, 0),
(26210, 26220, 'author', 'en', '◖_/¯❻❻❻◢¯\\_◖ :: Pokemon hack and cheats PokeCoins | AndroiD/iOS', 1771, 1, 1468428352, 1468428352, 0, 0, 0, 0, 0, 0),
(26211, 26221, 'author', 'en', '♢(GENERATOR)♤ 13:07:2016 :: Pokemon HAck Grati◁s hack POkeC*oins, PokeBalls | iOS-Android', 1771, 1, 1468428355, 1468428355, 0, 0, 0, 0, 0, 0),
(26212, 26222, 'author', 'en', '♇|¯\\_ENGINE_/¯|♇ 13:07:2016 :: Pokemon ch.eats th|at actually work generate PokeCoins, PokeBalls', 1771, 1, 1468428361, 1468428361, 0, 0, 0, 0, 0, 0),
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(26214, 26224, 'author', 'en', '❐❾❾❾❐ :: Pokemon hack a☺ndroid h.a.c.k. PokeCoins, PokeBaLls', 1771, 1, 1468428378, 1468428378, 0, 0, 0, 0, 0, 0),
(26215, 26225, 'author', 'en', '○#onl▽ine◌ 13-07-2016 :: Pokemon cheat coD❆E e;ntry gEnerate PokeCoiNs | Android/iOS', 1771, 1, 1468428380, 1468428380, 0, 0, 0, 0, 0, 0),
(26216, 26226, 'author', 'en', '✸(*(❹❹❹)*) :: Pokemon hack no root hack PokeCoins', 1771, 1, 1468428383, 1468428383, 0, 0, 0, 0, 0, 0),
(26217, 26227, 'author', 'en', '✬|_/¯❶❶❶¯\\_| :: Code de Triche Pokemon hack PokeBalls☏ | A☄ndroid', 1771, 1, 1468428399, 1468428399, 0, 0, 0, 0, 0, 0),
(26218, 26228, 'author', 'en', '✭[!cheat!] 13:07:2016 :: Pokemon Hack No Root ', 1771, 1, 1468428399, 1468428399, 0, 0, 0, 0, 0, 0),
(26219, 26229, 'author', 'en', '▤[![�!�!�!�!�!�!�!�!�!]!] ::  PokeCoins, PokeBalls', 1771, 1, 1468428404, 1468428404, 0, 0, 0, 0, 0, 0),
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(26223, 26233, 'author', 'en', '▨{~{GENERAT.OR}~} :: Pokemon ha✩ck no jailbreak Poke\\Balls', 1771, 1, 1468428423, 1468428423, 0, 0, 0, 0, 0, 0),
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(26225, 26235, 'author', 'en', '✱�..�❼❼ :: Pokemon cheats a/pk free PokeCoi,ns, PokeBalLs | iOS-And☚roid', 1771, 1, 1468428432, 1468428432, 0, 0, 0, 0, 0, 0),
(26226, 26236, 'author', 'en', '❧###2016 :: Pokemon Cheats ', 1771, 1, 1468428434, 1468428434, 0, 0, 0, 0, 0, 0),
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(26229, 26239, 'author', 'en', '✶!1,3,-,0,7,-,2,0,1,6, :: Pokemon cheats iph|on❃e generate PokeCoins | And☶roid/iOS', 1771, 1, 1468428449, 1468428449, 0, 0, 0, 0, 0, 0),
(26230, 26240, 'author', 'en', '◯###❶✳❶❶ :: Pokemon tIps advanced free P!okeBalls | iOS-Android', 1771, 1, 1468428456, 1468428456, 0, 0, 0, 0, 0, 0),
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(26232, 26242, 'author', 'en', 'Soccer~:W atch Atlético Huila vs Cortuluá L ive S treaming Full Coverage...', 1820, 1, 1468428461, 1468428461, 0, 0, 0, 0, 0, 0),
(26233, 26243, 'author', 'en', 'Soccer~:W atch Jaguares de Córdoba vs Once Caldas L ive S treaming Full Coverage...', 1820, 1, 1468428462, 1468428462, 0, 0, 0, 0, 0, 0),
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(26251, 26261, 'author', 'en', '♘#❽❽❽♘ :: Pokemon cheats android PokeBalls | Android', 1771, 1, 1468428489, 1468428489, 0, 0, 0, 0, 0, 0),
(26252, 26262, 'author', 'en', 'Soccer~:W atch Botafogo PB vs Ceará L ive S treaming Full Coverage...', 1820, 1, 1468428490, 1468428490, 0, 0, 0, 0, 0, 0),
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(26258, 26268, 'author', 'en', 'Soccer~:W atch Deportivo Cuenca vs Delfin L ive S treaming Full Coverage...', 1820, 1, 1468428499, 1468428499, 0, 0, 0, 0, 0, 0),
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(26263, 26273, 'author', 'en', 'Soccer~:W atch Al Ahly Shendi vs Hilal Obayed L ive S treaming Full Coverage...', 1820, 1, 1468428509, 1468428509, 0, 0, 0, 0, 0, 0),
(26264, 26274, 'author', 'en', 'Soccer~:W atch Al Rabta Kosti vs Khartoum 3 L ive S treaming Full Coverage...', 1820, 1, 1468428512, 1468428512, 0, 0, 0, 0, 0, 0),
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(26266, 26276, 'author', 'en', '☓[*[❽❽❽]*]☓ :: no survey hack google play Pokemon  ', 1771, 1, 1468428526, 1468428526, 0, 0, 0, 0, 0, 0),
(26267, 26277, 'author', 'en', '◈[![❺❺❺]!]◉ :: Pokemon hack ifile PokeCoins | 2016', 1771, 1, 1468428526, 1468428526, 0, 0, 0, 0, 0, 0),
(26268, 26278, 'author', 'en', '❃{!❶❶❶!}❅ :: Pokemon cheats iphone PokeBalls | Android/iOS', 1771, 1, 1468428531, 1468428531, 0, 0, 0, 0, 0, 0),
(26269, 26279, 'author', 'en', '✨(!meg♂ahack!)✨ 13:07:2016 :: Pokemon cheats dOwn◵load ', 1771, 1, 1468428535, 1468428535, 0, 0, 0, 0, 0, 0),
(26270, 26280, 'author', 'en', '▼(!(❾�☙��❾)!)▽ :: Pokemon cheats and hacks PokeBall,s | Android', 1771, 1, 1468428542, 1468428542, 0, 0, 0, 0, 0, 0),
(26271, 26281, 'author', 'en', '❒(❾❾❾) :: Pokemon cheAts ios PokeCoins | 2016', 1771, 1, 1468428564, 1468428564, 0, 0, 0, 0, 0, 0),
(26272, 26282, 'author', 'en', '◾¯\\_❷❷❷_/¯◿ :: Pokemon Hack Cheat Juwelen DeutSch! free PokeCoins | Android/iOS', 1771, 1, 1468428566, 1468428566, 0, 0, 0, 0, 0, 0),
(26273, 26283, 'author', 'en', '▣[[2016]]▤ :: Pokemon pirater telecharger hack PokeBAlls | Android/iOS', 1771, 1, 1468428566, 1468428566, 0, 0, 0, 0, 0, 0),
(26274, 26284, 'author', 'en', '{*GENER*ATOR*} 13\\/07/2016 :: Pokemon Hack Gratis hack PokeCoins', 1771, 1, 1468428571, 1468428571, 0, 0, 0, 0, 0, 0),
(26275, 26285, 'author', 'en', '✧((2016))✨ 13-07-2016 :: Pokemon Juwelen H.a.c.k. generate Po!keCoins, PokeBalls', 1771, 1, 1468428587, 1468428587, 0, 0, 0, 0, 0, 0),
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(26279, 26289, 'author', 'en', '♵❼❼❼♵ :: Pokemon hack and cheats free PokeBalls | Android/i.OS', 1771, 1, 1468428598, 1468428598, 0, 0, 0, 0, 0, 0),
(26280, 26290, 'author', 'en', '❎[![❼❼❼]!]❏ :: Pokemon tips an♒d, tricks hack PokeCoins', 1771, 1, 1468428611, 1468428611, 0, 0, 0, 0, 0, 0),
(26281, 26291, 'author', 'en', '❃{GENERATOR} 13/07/2016 :: Pokemon Glitch generate PokeBalls', 1771, 1, 1468428612, 1468428612, 0, 0, 0, 0, 0, 0),
(26282, 26292, 'author', 'en', '✒!❼❼❼✒ :^: Astuces Pour Pokemon hack PokeCoins, PokeBAll.s | Android/iOS', 1771, 1, 1468428614, 1468428614, 0, 0, 0, 0, 0, 0),
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(26286, 26296, 'author', 'en', '❙(*(❺❺❺)*)❚ :: Pokemon triCheurs free PokeCoIns, P☬okeBal♊l▥s | iOS-Android', 1771, 1, 1468428636, 1468428636, 0, 0, 0, 0, 0, 0),
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(26290, 26300, 'author', 'en', '❁[!❾❾❾☼!]❁ :: Pokemon cheats for ipad generate PokeBalls | Android', 1771, 1, 1468428670, 1468428670, 0, 0, 0, 0, 0, 0),
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(26301, 26311, 'author', 'en', '♰(*(07:13:2016)*)♰ :: Pokemon cheat that works free PokeCoins, PokeBalls |.. 2016', 1771, 1, 1468428734, 1468428734, 0, 0, 0, 0, 0, 0),
(26302, 26312, 'author', 'en', '◣~!~❼❼❼◣ :: Pokemon hack android generate PokeBalls | 2016☼', 1771, 1, 1468428734, 1468428734, 0, 0, 0, 0, 0, 0),
(26303, 26313, 'author', 'en', '♉(~❻❻❻~)♊ :: how to hack Pokemon PokeCoins | Andro.id/iOS', 1771, 1, 1468428736, 1468428736, 0, 0, 0, 0, 0, 0),
(26304, 26314, 'author', 'en', '❓{*❶❶❶*}❓ :: Pokemon hack no dow:nload ', 1771, 1, 1468428742, 1468428742, 0, 0, 0, 0, 0, 0),
(26305, 26315, 'author', 'en', '☉{!{❻❻�*�}!}☉ :: Pokemon ipa hack PokeBalls | Android', 1771, 1, 1468428753, 1468428753, 0, 0, 0, 0, 0, 0),
(26306, 26316, 'author', 'en', '❰{!�◝��❹�.��!} :: Pokemon hack ifile free PoKeCo✧ins, PokeBalls | Android', 1771, 1, 1468428757, 1468428757, 0, 0, 0, 0, 0, 0),
(26307, 26317, 'author', 'en', '▱(!(❻❻❻)!) :: Pokemon tips tricks free PokeBalls | 2016', 1771, 1, 1468428758, 1468428758, 0, 0, 0, 0, 0, 0),
(26308, 26318, 'author', 'en', '™{~{hack}~} 13.07.2016 :: Pokemon hack work PokeBalls | Android/iOS', 1771, 1, 1468428761, 1468428761, 0, 0, 0, 0, 0, 0),
(26309, 26319, 'author', 'en', '☕(*(❽❽❽)*)☖ :: Pokemon cheat engine gener/ate PokeBalls | iOS-Android', 1771, 1, 1468428764, 1468428764, 0, 0, 0, 0, 0, 0),
(26310, 26320, 'author', 'en', '△{*{❸❸❸}*}△ :: how to h.a.c.k. Pokemon hack PokeBalls', 1771, 1, 1468428775, 1468428775, 0, 0, 0, 0, 0, 0),
(26311, 26321, 'author', 'en', '▪❺❺❺▪ :: Pokemon cheats for android PokeCoIns | Andro..id/iOS', 1771, 1, 1468428783, 1468428783, 0, 0, 0, 0, 0, 0),
(26312, 26322, 'author', 'en', '☸|_/¯❸�♉�❸¯\\_|☸ :: Pokemon hack tool hack PokeBalls | Android/iOS', 1771, 1, 1468428784, 1468428784, 0, 0, 0, 0, 0, 0),
(26313, 26323, 'author', 'en', '☉{*{online}*}☊ :: Pokemon hack safe generate PokeCoins', 1771, 1, 1468428805, 1468428805, 0, 0, 0, 0, 0, 0),
(26314, 26324, 'author', 'en', '♑[*[❽❽❽]*]♒ :: Pokemon hack tool ', 1771, 1, 1468428807, 1468428807, 0, 0, 0, 0, 0, 0),
(26315, 26325, 'author', 'en', '(~❸❸�❁��~) :: Pokemon hack jaibreak free PokeCoins', 1771, 1, 1468428807, 1468428807, 0, 0, 0, 0, 0, 0),
(26316, 26326, 'author', 'en', '❙#❾�^�❾❚ :: Pokemon Generator gen❒erAte PokeBalls | Android/iOS', 1771, 1, 1468428820, 1468428820, 0, 0, 0, 0, 0, 0),
(26317, 26327, 'author', 'en', '▮❷❷❷ ::  hack Po.keBalls', 1771, 1, 1468428827, 1468428827, 0, 0, 0, 0, 0, 0),
(26318, 26328, 'author', 'en', '♵❼❼�☔�♵ :: Pokemon hack mac free PokeCoins, PokeBa.lls | 2016', 1771, 1, 1468428837, 1468428837, 0, 0, 0, 0, 0, 0),
(26319, 26329, 'author', 'en', '◤!MA✱Y◥ 13-07-2016 :: Pokemon cheats free PokeCoins, PokeBalls❁ | 2016', 1771, 1, 1468428838, 1468428838, 0, 0, 0, 0, 0, 0),
(26320, 26330, 'author', 'en', '◒[hackengine] 13:07:2016 :: Pokemon Online HacK PokeCoins, PokeBalls | Android', 1771, 1, 1468428839, 1468428839, 0, 0, 0, 0, 0, 0),
(26321, 26331, 'author', 'en', '▯((❹❹❹)) :: Pokemon mod PokeBallS | An:droid/iOS◫', 1771, 1, 1468428841, 1468428841, 0, 0, 0, 0, 0, 0),
(26322, 26332, 'author', 'en', '◾|_/¯ENGINE¯\\_| 13:07:2016 :: Code: de Triche Pokemon generate PokeBalls | iOS-And^roid', 1771, 1, 1468428853, 1468428853, 0, 0, 0, 0, 0, 0),
(26323, 26333, 'author', 'en', '♀(!❹❹❹!)♀ :: Pokemon cheats reviews ', 1771, 1, 1468428880, 1468428880, 0, 0, 0, 0, 0, 0),
(26324, 26334, 'author', 'en', '✰[~❽❽❽~]✰ :: Pokemon cheats for i☠phOne fr☽ee PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468428882, 1468428882, 0, 0, 0, 0, 0, 0),
(26325, 26335, 'author', 'en', '◯{!❸❸❸!}◰ :: Pokemon hack activation code PokeBalls | Andr◀oid/iOS', 1771, 1, 1468428882, 1468428882, 0, 0, 0, 0, 0, 0),
(26326, 26336, 'author', 'en', '▧[!❹❹❹!]▧ :: Pokemon apk cheats free PokeCoins | iOS-And.roid', 1771, 1, 1468428883, 1468428883, 0, 0, 0, 0, 0, 0),
(26327, 26337, 'author', 'en', '◻(!MOD!)◼ 13/07/2016 :: Pokemon hack for iphone generate PokeBalls | 2○016', 1771, 1, 1468428902, 1468428902, 0, 0, 0, 0, 0, 0),
(26328, 26338, 'author', 'en', '✓[~[july]~]✗ :: Pokemon hack Ifunbox generate PokeCoins | Android/iOS', 1771, 1, 1468428906, 1468428906, 0, 0, 0, 0, 0, 0),
(26329, 26339, 'author', 'en', '❋{*{gethack}*}❋ 13-07▭-2016 :: Pokemon hack android g:enerate PokeCoins, P|o|k|e|B|a|l|l|s| | iO❇S-Android', 1771, 1, 1468428909, 1468428909, 0, 0, 0, 0, 0, 0),
(26330, 26340, 'author', 'en', '♼[~❽❽❽~]♼ :: Pokemon hack fOr android free PokeBalls | 2016', 1771, 1, 1468428910, 1468428910, 0, 0, 0, 0, 0, 0),
(26331, 26341, 'author', 'en', '❋{*❹◮❹❹*}❌ :: Hack Para Pokemon generate PokeCoins, P..o..k..e..B..a..l..l..s.. | 2016', 1771, 1, 1468428917, 1468428917, 0, 0, 0, 0, 0, 0),
(26332, 26342, 'author', 'en', '☔{!❾❾❾!}☕ :: Pokemon cheats fOr ipHone f;r;e;e; PokeBalls', 1771, 1, 1468428928, 1468428928, 0, 0, 0, 0, 0, 0),
(26333, 26343, 'author', 'en', '♢{!{HACKPACK}!}♤ 13.07.2016 :: Pokemon hack online hack PokeCoins, PokeBalls | 2..016', 1771, 1, 1468428934, 1468428934, 0, 0, 0, 0, 0, 0),
(26334, 26344, 'author', 'en', '▻[~engine~] 13/07/2016 :: Pokemon Hack iPhon,e iPad Android free PokeCoins,\\ Pok:eBalls☍ | A,n,d,r,o,i,d,/,i,O,S,', 1771, 1, 1468428936, 1468428936, 0, 0, 0, 0, 0, 0),
(26335, 26345, 'author', 'en', '☳$❹❹❹☴ :: Pokemon che◹ats online hack PokeBalls | 2016', 1771, 1, 1468428938, 1468428938, 0, 0, 0, 0, 0, 0),
(26336, 26346, 'author', 'en', '◰[❷❷❷]◰ :: Pokemon Online Generator ', 1771, 1, 1468428940, 1468428940, 0, 0, 0, 0, 0, 0),
(26337, 26347, 'author', 'en', '☵(!❽❽❽!)☵ :: Pokemon hack codes ', 1771, 1, 1468428967, 1468428967, 0, 0, 0, 0, 0, 0),
(26338, 26348, 'author', 'en', '➤(!Hacktool!)➤ 13/07☫/2016 :: Pokemon hack online free PokeBalls | AnDroid/iOS', 1771, 1, 1468428990, 1468428990, 0, 0, 0, 0, 0, 0),
(26339, 26349, 'author', 'en', '❓(!engine!)❔ 13/07/201◑6 :: Pokemon h☫ack ipad PokeCoins, PokeBalls | A.n.d.r.o.i.d./.i.O.S.', 1771, 1, 1468428992, 1468428992, 0, 0, 0, 0, 0, 0),
(26340, 26350, 'author', 'en', '✩(!(❶❶�❇�☼�)!) :: Pokemon Hack Gratis hack❟ PokeCoins, Pok♊eBalls', 1771, 1, 1468428993, 1468428993, 0, 0, 0, 0, 0, 0),
(26341, 26351, 'author', 'en', '▹[!❽❽❽!]▹ :: Pokemon hack no sur.vey  PokeCoins, PokeBalls', 1771, 1, 1468428994, 1468428994, 0, 0, 0, 0, 0, 0),
(26342, 26352, 'author', 'en', '▰{!{❶✶❶❶}!}▱ :: Pokemon cheat engine hack PokeCoins', 1771, 1, 1468428994, 1468428994, 0, 0, 0, 0, 0, 0),
(26343, 26353, 'author', 'en', '☯(~❾�!�❾~)☯ :: Pokemon hack software ', 1771, 1, 1468429011, 1468429011, 0, 0, 0, 0, 0, 0),
(26344, 26354, 'author', 'en', '♸(!07/13/2016!) :: Pokemon tricheurs free PokeCoins |! A☯ndroid', 1771, 1, 1468429015, 1468429015, 0, 0, 0, 0, 0, 0),
(26345, 26355, 'author', 'en', '☳❺❺❺☴ :\\: Hack Para Pokemon ', 1771, 1, 1468429018, 1468429018, 0, 0, 0, 0, 0, 0),
(26346, 26356, 'author', 'en', '☌¯\\_mod_/¯☌ 13-0,7-2016 :: Hack PaRa Pokemon PokeCoins | 2016', 1771, 1, 1468429018, 1468429018, 0, 0, 0, 0, 0, 0),
(26347, 26357, 'author', 'en', '☮(*cheat*)☮ 13-07-2016 :: Pokemon hack that works ', 1771, 1, 1468429020, 1468429020, 0, 0, 0, 0, 0, 0),
(26348, 26358, 'author', 'en', '▴#❷❷❷▵ :: Pokemon cheats ios PokeCoins, Po☺keBalls', 1771, 1, 1468429038, 1468429038, 0, 0, 0, 0, 0, 0),
(26349, 26359, 'author', 'en', '▷((engine))▷ :: Pokemon Astuce free PokeCoins | Android/iOS', 1771, 1, 1468429040, 1468429040, 0, 0, 0, 0, 0, 0),
(26350, 26360, 'author', 'en', '♔~!~❹❹❹ :: Pokemon hack iphone hack PokeCoins | iOS-ANdroid', 1771, 1, 1468429042, 1468429042, 0, 0, 0, 0, 0, 0),
(26351, 26361, 'author', 'en', '☐{{❻❻❻}}☑ :: Pokemon cheats for iphone free PokeBalls | 2/016', 1771, 1, 1468429043, 1468429043, 0, 0, 0, 0, 0, 0),
(26352, 26362, 'author', 'en', '☲(~HACK~)☳ 13/07/2016 :: Pokemon c^h^e^a^t^s^ for iphoN▤e PokeCoins', 1771, 1, 1468429054, 1468429054, 0, 0, 0, 0, 0, 0),
(26353, 26363, 'author', 'en', '❙#GENERATOR❙ :: Pokemon a/pk mega mod ', 1771, 1, 1468429059, 1468429059, 0, 0, 0, 0, 0, 0),
(26354, 26364, 'author', 'en', '❀(~❼❼❼~)❀ ::❋ Pokemon hack activation c/o/d/e/ hack PokeCoins | 2016', 1771, 1, 1468429062, 1468429062, 0, 0, 0, 0, 0, 0),
(26355, 26365, 'author', 'en', '❕[~MEGAHACK~] :: Pokemon Hack iPhone iPad Android PokeBalls | An✷dro♻i.d/iOS', 1771, 1, 1468429062, 1468429062, 0, 0, 0, 0, 0, 0),
(26356, 26366, 'author', 'en', '◳_/¯MAY¯\\_ :: how to hack Pokemon generate PokeCoins | And✲roid/iOS', 1771, 1, 1468429064, 1468429064, 0, 0, 0, 0, 0, 0),
(26357, 26367, 'author', 'en', '☸[!❻❻❻!]☸ :: Pokemon Online Hack ', 1771, 1, 1468429074, 1468429074, 0, 0, 0, 0, 0, 0),
(26358, 26368, 'author', 'en', '▢~!~❾❾❾▣ :: Pokemon hack tele♁charger freE PokeCoi.ns, PokeBalls | Android', 1771, 1, 1468429078, 1468429078, 0, 0, 0, 0, 0, 0),
(26359, 26369, 'author', 'en', '♀[h*ac♌ked]♀ :: Pokemon chea♁ts and tips ', 1771, 1, 1468429084, 1468429084, 0, 0, 0, 0, 0, 0),
(26360, 26370, 'author', 'en', '◾❼❼❼◿ :: Pokemon Hacked apk PokeCoins | Android', 1771, 1, 1468429086, 1468429086, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(26361, 26371, 'author', 'en', '❎((❺❺❺)) ::^ Pokemon cheat code entry hack PokeCoins◷, Pok.eBa.lLs | iOS-Android', 1771, 1, 1468429090, 1468429090, 0, 0, 0, 0, 0, 0),
(26362, 26372, 'author', 'en', '◬(!(HACKCHEATS)!)◬ 13-07-2016 :: Pokemon hack no survey  hack PokeBalls | Android', 1771, 1, 1468429094, 1468429094, 0, 0, 0, 0, 0, 0),
(26363, 26373, 'author', 'en', '◜❶❶❶ :: Pokemon cheats android generate PokeCoins, PokeBalls♔ | 2016', 1771, 1, 1468429117, 1468429117, 0, 0, 0, 0, 0, 0),
(26364, 26374, 'author', 'en', '♪[*[❹❹❹]*]♪ :: Code de Triche Pokemon ', 1771, 1, 1468429122, 1468429122, 0, 0, 0, 0, 0, 0),
(26365, 26375, 'author', 'en', '♬{~{mod}~}♭ 13/07/2016 :✰: Pokemon cheats ipad g|enerate PokeBalls | Android', 1771, 1, 1468429126, 1468429126, 0, 0, 0, 0, 0, 0),
(26366, 26376, 'author', 'en', '✤#❽❽❽✥ :: Pokemon Hacken free PokeCoins', 1771, 1, 1468429126, 1468429126, 0, 0, 0, 0, 0, 0),
(26367, 26377, 'author', 'en', '❺❺❺ :: Pokemon Cheats ', 1771, 1, 1468429130, 1468429130, 0, 0, 0, 0, 0, 0),
(26368, 26378, 'author', 'en', '✡{*❶❶❶*}✡ :: Pokemon cheat download generate PokeCoins', 1771, 1, 1468429137, 1468429137, 0, 0, 0, 0, 0, 0),
(26369, 26379, 'author', 'en', '☺[*[MAY2016]*]☺ :: Pokemon Hack hack PokeCoins, P.okeBalls | iOS-Android', 1771, 1, 1468429150, 1468429150, 0, 0, 0, 0, 0, 0),
(26370, 26380, 'author', 'en', '♭(~(online)~)♭ 13/07/201\\6 :: Pokemon Cheats ios Poke♵Coins | iOS-An*droid', 1771, 1, 1468429150, 1468429150, 0, 0, 0, 0, 0, 0),
(26371, 26381, 'author', 'en', '❎$❹❹❹❎ :: Pokemon Triche hack POkeBa▷lls | 2016', 1771, 1, 1468429150, 1468429150, 0, 0, 0, 0, 0, 0),
(26372, 26382, 'author', 'en', '☳|¯\\_HACKGENERAT\\OR_/¯|☳ :: Pokemon cheat code entry generate PokeCoinS, PokeBalls | Android/iOS', 1771, 1, 1468429153, 1468429153, 0, 0, 0, 0, 0, 0),
(26373, 26383, 'author', 'en', '◸(*(❻❻❻)*) :: Pokemon hack tool download ', 1771, 1, 1468429176, 1468429176, 0, 0, 0, 0, 0, 0),
(26374, 26384, 'author', 'en', '♛(!2016!)♜ 13.07.2016 :: Pokemon hack no download  free PokeCoins | Android', 1771, 1, 1468429177, 1468429177, 0, 0, 0, 0, 0, 0),
(26375, 26385, 'author', 'en', '☿[*[❻❻❻]*] :: Pokemon Mod Apk ', 1771, 1, 1468429179, 1468429179, 0, 0, 0, 0, 0, 0),
(26376, 26386, 'author', 'en', '♲{!❾❾❾!}♲ :: Pokemon Hack No Root generate PokeBalls | AndrOid/iOS', 1771, 1, 1468429180, 1468429180, 0, 0, 0, 0, 0, 0),
(26377, 26387, 'author', 'en', '◆{!{megahack}!} 13-07-2016 :: Pokemon APK IPA Mod generate PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468429183, 1468429183, 0, 0, 0, 0, 0, 0),
(26378, 26388, 'author', 'en', '☿[![❺❺❺]!]☀ :: Pokemon Juwelen H^ack hack PokeCoins, PokeBalls | Android/iO.S', 1771, 1, 1468429199, 1468429199, 0, 0, 0, 0, 0, 0),
(26379, 26389, 'author', 'en', '○❹❹❹◌ :: Pokemon Hack No Survey PokeCoins, PokeBalls | Andr♮oid/iOS', 1771, 1, 1468429199, 1468429199, 0, 0, 0, 0, 0, 0),
(26380, 26390, 'author', 'en', '☺(*(�★�❸❸)*)☻ :: cheats for Pokemon hack PokeCoins, PokeBalls |. 2016', 1771, 1, 1468429203, 1468429203, 0, 0, 0, 0, 0, 0),
(26381, 26391, 'author', 'en', '☄(❻❻◈❻)☄ :: Pokemon cheats ipad.◾ PokeCoins, PokeBalls |: i♢OS-Android', 1771, 1, 1468429204, 1468429204, 0, 0, 0, 0, 0, 0),
(26382, 26392, 'author', 'en', '♶!❺�♹�❺ :: Pokemon APK IPA Mod free PokeBalls', 1771, 1, 1468429204, 1468429205, 0, 0, 0, 0, 0, 0),
(26383, 26393, 'author', 'en', '✣(*(07/13/2016)*) :: Pokemon Hack Deutsch free P!o!k!e!C!o!i!n!s!', 1771, 1, 1468429220, 1468429220, 0, 0, 0, 0, 0, 0),
(26384, 26394, 'author', 'en', '▢|¯\\_❹❹❹_/¯| :: Pokemon tips﨏 and tricks ', 1771, 1, 1468429220, 1468429220, 0, 0, 0, 0, 0, 0),
(26385, 26395, 'author', 'en', '☃[~❷❷❷~]☃ :: how to get h..a..c..k.. tool Pokemon hack PokeCoins, PokeBalls', 1771, 1, 1468429225, 1468429225, 0, 0, 0, 0, 0, 0),
(26386, 26396, 'author', 'en', '❜|¯\\_❻❻❻_/¯| :: Pokemon Mod Apk free PokeBalls |/ Android', 1771, 1, 1468429226, 1468429226, 0, 0, 0, 0, 0, 0),
(26387, 26397, 'author', 'en', '☃(!(❽❽❽)!)☄ :: Pokemon Hack Cheat Juwelen Deutsch! generate. PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468429227, 1468429227, 0, 0, 0, 0, 0, 0),
(26388, 26398, 'author', 'en', '❰(*HACKEN^GINE*)❰ :: Pokemon hack mod♰ apk PokeCoins | Android', 1771, 1, 1468429248, 1468429248, 0, 0, 0, 0, 0, 0),
(26389, 26399, 'author', 'en', '♔(~❸❸❸~) :: Pokemon cheats/ android hack PokeCoins | Android', 1771, 1, 1468429268, 1468429268, 0, 0, 0, 0, 0, 0),
(26390, 26400, 'author', 'en', '◍(~(�❀��❾❾)~)◎ :: Pokemon Hack No Survey free P.okeCoins', 1771, 1, 1468429269, 1468429269, 0, 0, 0, 0, 0, 0),
(26391, 26401, 'author', 'en', '♴[~gethack~] 13:.07:2016 :: Pokemon Hack P\\o\\k\\e\\C\\o\\i\\n\\s\\,\\ PokeBalls', 1771, 1, 1468429269, 1468429269, 0, 0, 0, 0, 0, 0),
(26392, 26402, 'author', 'en', '◮(!�◵�❷❷!)◮ :: Pokemon cheats and hacks ', 1771, 1, 1468429275, 1468429275, 0, 0, 0, 0, 0, 0),
(26393, 26403, 'author', 'en', '❏july❏ 13-07-2016 :.:. Pokemon hack mod free PokeCoins | 2016', 1771, 1, 1468429275, 1468429275, 0, 0, 0, 0, 0, 0),
(26394, 26404, 'author', 'en', '☮(~(generator)~)☮ 13-07-2016 :.:.  ', 1771, 1, 1468429299, 1468429299, 0, 0, 0, 0, 0, 0),
(26395, 26405, 'author', 'en', '☯(!(�◺�❺❺)!)☯ :: Pokemon cheaTs real PokeCoins | Android', 1771, 1, 1468429299, 1468429299, 0, 0, 0, 0, 0, 0),
(26396, 26406, 'author', 'en', '♛[![hack]!] 13.07.2016 :: Pokemon Pirater ', 1771, 1, 1468429303, 1468429303, 0, 0, 0, 0, 0, 0),
(26397, 26407, 'author', 'en', '✾[!CHEAT!] 13/07/2016 :;:; Pokemon cheaTs iphone POkeBalls | 2016', 1771, 1, 1468429303, 1468429303, 0, 0, 0, 0, 0, 0),
(26398, 26408, 'author', 'en', '◆$❾❾❾ :: Pokemon Juwelen Cheat PokeBalls | 2016', 1771, 1, 1468429313, 1468429313, 0, 0, 0, 0, 0, 0),
(26399, 26409, 'author', 'en', '♗[![GENERATOR]!]♗ 13/07/2.016 :: Pokemon ha﨏c.k app PokeBalls', 1771, 1, 1468429320, 1468429320, 0, 0, 0, 0, 0, 0),
(26400, 26410, 'author', 'en', '▶{!{07-13-2016}!} :: Pokemon Glitch and Gli◆tches. free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468429321, 1468429321, 0, 0, 0, 0, 0, 0),
(26401, 26411, 'author', 'en', '▿{{❾❾❾}}◠ :: Pokemon cheats for mobile hack PokeCoins, PokeBalls |, iOS-Android', 1771, 1, 1468429325, 1468429325, 0, 0, 0, 0, 0, 0),
(26402, 26412, 'author', 'en', '✫{~❷❷❷~}✬ :: Pokemon cheat download hacK PokeCoins, PokeBalls | 2016', 1771, 1, 1468429327, 1468429327, 0, 0, 0, 0, 0, 0),
(26403, 26413, 'author', 'en', '❉[*[❼❼;❼]*]❊ :: Pokemon cheats for ipad hac☙k PokeBaLl,s | Android/iOS', 1771, 1, 1468429336, 1468429336, 0, 0, 0, 0, 0, 0),
(26404, 26414, 'author', 'en', '☓[*❾❾❾*]☓ :: Pokemon hack no root ', 1771, 1, 1468429342, 1468429342, 0, 0, 0, 0, 0, 0),
(26405, 26415, 'author', 'en', '♗[![CHEAT]!]♘ 13-07-2016 :: Pokemon hack moD apk ', 1771, 1, 1468429344, 1468429344, 0, 0, 0, 0, 0, 0),
(26406, 26416, 'author', 'en', '◴[~❶❶❶~] :: Pokemon cheats for ipad PokeCoIns', 1771, 1, 1468429347, 1468429347, 0, 0, 0, 0, 0, 0),
(26407, 26417, 'author', 'en', '▽~!~online▾ 13//07/2016 :: Pokemon ipa hack PokeCoins | Android/iOS', 1771, 1, 1468429348, 1468429348, 0, 0, 0, 0, 0, 0),
(26408, 26418, 'author', 'en', '®{~13:07:2016~} :: Pokemon hacK andr^oid free PokeCoins, PokeBalls | iOS-And◪roid', 1771, 1, 1468429358, 1468429358, 0, 0, 0, 0, 0, 0),
(26409, 26419, 'author', 'en', '♡{❻/❻�◆��}♡ :: Pokemon hack no downlOad PokeCoin.s, PokeBall☱s', 1771, 1, 1468429364, 1468429364, 0, 0, 0, 0, 0, 0),
(26410, 26420, 'author', 'en', '◃{!HACKENGINE!}◄ 13.07...2016 :: Pokemon cheats ios PokeCoins | iOS-And☘r.oid', 1771, 1, 1468429365, 1468429365, 0, 0, 0, 0, 0, 0),
(26411, 26421, 'author', 'en', '❑~!~❻❻❻❒ :: Pokemon hack iphone PokeBalls', 1771, 1, 1468429369, 1468429369, 0, 0, 0, 0, 0, 0),
(26412, 26422, 'author', 'en', '❃!13.07.2016❃ :: Pokemon hacked apk hack PokeCoins, | Android/iOS', 1771, 1, 1468429371, 1468429371, 0, 0, 0, 0, 0, 0),
(26413, 26423, 'author', 'en', '♯(~(❾❾❾)~)♯ :: Pokemon hack downloAd ', 1771, 1, 1468429383, 1468429383, 0, 0, 0, 0, 0, 0),
(26414, 26424, 'author', 'en', '◐(~❷❷❷~)◐ :: Hack Para Pokemon PokeCoins, PokeBalls', 1771, 1, 1468429407, 1468429407, 0, 0, 0, 0, 0, 0),
(26415, 26425, 'author', 'en', '☮¯\\_07:13◈:2016_/¯☮ :♬: Pokemon cheats and tips. ', 1771, 1, 1468429410, 1468429410, 0, 0, 0, 0, 0, 0),
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(26418, 26428, 'author', 'en', '◚[~[❾❾❾]~] :: Pokemon hack safe hack PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468429418, 1468429418, 0, 0, 0, 0, 0, 0),
(26419, 26429, 'author', 'en', '☗CHEAT☗ :: Pokemon Hack Tool APK IPA PokeCoinS | 2016', 1771, 1, 1468429431, 1468429431, 0, 0, 0, 0, 0, 0),
(26420, 26430, 'author', 'en', '♒[~[generato✩r]~]♒ 13:07:2016 :: Pokemon Glitch and Glitches ', 1771, 1, 1468429436, 1468429436, 0, 0, 0, 0, 0, 0),
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(26422, 26432, 'author', 'en', '◃[![HACKENGINE]!]◃ 13/07/2016 :.: Pokemon cheat apk free PokeCoins', 1771, 1, 1468429440, 1468429440, 0, 0, 0, 0, 0, 0),
(26423, 26433, 'author', 'en', '☓$07-13-2016☓ :: Pokemon hack work ', 1771, 1, 1468429450, 1468429450, 0, 0, 0, 0, 0, 0),
(26424, 26434, 'author', 'en', '✴[FULLHACK]✵ 13.07.2016 :: Pokemon cheats oNline ', 1771, 1, 1468429453, 1468429453, 0, 0, 0, 0, 0, 0),
(26425, 26435, 'author', 'en', '◓¯\\_❷❷❷_/¯◔ :: Pokemon cheaTs* Apk ', 1771, 1, 1468429457, 1468429457, 0, 0, 0, 0, 0, 0),
(26426, 26436, 'author', 'en', '◩#M☕EGAHACK◪ 13-07-2016 :: Pokemon hack activation cOde hack PokeCoins', 1771, 1, 1468429460, 1468429460, 0, 0, 0, 0, 0, 0),
(26427, 26437, 'author', 'en', '◈{!{2016}!}◉ 13:07:2016 :: Pokemon cheAt review ', 1771, 1, 1468429461, 1468429461, 0, 0, 0, 0, 0, 0),
(26428, 26438, 'author', 'en', '♆¯\\_hack_/¯♇ 13:07:2016 :: Pokemon hack generator ', 1771, 1, 1468429485, 1468429485, 0, 0, 0, 0, 0, 0),
(26429, 26439, 'author', 'en', '❖(!(❼�♸��❼)!)❗ :: Pokemon hack ifunbox ', 1771, 1, 1468429488, 1468429488, 0, 0, 0, 0, 0, 0),
(26430, 26440, 'author', 'en', '♥❹❹❹♦ :: Pokemon Hack Gratis PokeCoins, PokeBalls', 1771, 1, 1468429489, 1468429489, 0, 0, 0, 0, 0, 0),
(26431, 26441, 'author', 'en', '☳[~❺❺❺~]☴ :: Pokemon Hack Deutsch free PokeBalls | 2016', 1771, 1, 1468429489, 1468429489, 0, 0, 0, 0, 0, 0),
(26432, 26442, 'author', 'en', '◄(*(❾❾❾)*)◄ :: no s;urvey hack gooGle play Pokemon  free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468429491, 1468429491, 0, 0, 0, 0, 0, 0),
(26433, 26443, 'author', 'en', '◽❼❼❼◾ :: Pokemon hack tool online ', 1771, 1, 1468429506, 1468429506, 0, 0, 0, 0, 0, 0),
(26434, 26444, 'author', 'en', '☶(!(❻❻❻)!)☷ :: Pokemon hack codes free PokeBalls | iOS-Android', 1771, 1, 1468429512, 1468429512, 0, 0, 0, 0, 0, 0),
(26435, 26445, 'author', 'en', '✷[!july2016!]✸ :: Pokemon cheat tool♱ free PokeBalls | Android', 1771, 1, 1468429513, 1468429513, 0, 0, 0, 0, 0, 0),
(26436, 26446, 'author', 'en', '◇(*(hackpack)*) 13:07:2016 :: Pokemon haCk to▪ol generator PokeCoins, P|okeBalls | Androi✯d/iOS', 1771, 1, 1468429515, 1468429515, 0, 0, 0, 0, 0, 0),
(26437, 26447, 'author', 'en', '▽[07-13-2016]▽ :: Pokemon ipa hack ', 1771, 1, 1468429516, 1468429516, 0, 0, 0, 0, 0, 0),
(26438, 26448, 'author', 'en', '◸$❹❹❹◹ :: Pokemon Hack. Cheats Trainer free PokeCoins | Android', 1771, 1, 1468429536, 1468429536, 0, 0, 0, 0, 0, 0),
(26439, 26449, 'author', 'en', '☪(!❶❶❶!) :..: Pokemon Triche Gratuit ', 1771, 1, 1468429554, 1468429554, 0, 0, 0, 0, 0, 0),
(26440, 26450, 'author', 'en', '☯!❺❺❺✈ :: Pokemon a!p;k mega mod◪ free PokeCoins, PokeBAlls▲', 1771, 1, 1468429555, 1468429555, 0, 0, 0, 0, 0, 0),
(26441, 26451, 'author', 'en', '☛~!~❻❻�:�☕�☛ :: Pokemon Online Hack PokeCoins | Android', 1771, 1, 1468429558, 1468429558, 0, 0, 0, 0, 0, 0),
(26442, 26452, 'author', 'en', '♳$❾❾❾ :: Pokemon hack iphone no root ', 1771, 1, 1468429560, 1468429560, 0, 0, 0, 0, 0, 0),
(26443, 26453, 'author', 'en', '☣[~❺❺❺~]☤ ::♖ Pokemon Astuce free PokeBalls', 1771, 1, 1468429563, 1468429563, 0, 0, 0, 0, 0, 0),
(26444, 26454, 'author', 'en', '◁[hackengine] 13-07-2016 :: Pokemon hack mod apk hack PokeCOins, PokeBalls✮ | Android/iOS', 1771, 1, 1468429576, 1468429576, 0, 0, 0, 0, 0, 0),
(26445, 26455, 'author', 'en', '◧{!07.13.;☮2016!}◨ :: Pokemon hack i☲pad free PokeBalls | iOS-Android', 1771, 1, 1468429578, 1468429578, 0, 0, 0, 0, 0, 0),
(26446, 26456, 'author', 'en', '☗{~{❶❶❶}~} :: Pokemon cheat tool free P☟okeCoins', 1771, 1, 1468429586, 1468429586, 0, 0, 0, 0, 0, 0),
(26447, 26457, 'author', 'en', '◣_/¯HACKCHEATS¯\\_◤ 13-07-2016 :: Pokemon tips tricks ', 1771, 1, 1468429587, 1468429587, 0, 0, 0, 0, 0, 0),
(26448, 26458, 'author', 'en', '✬❻❻❻ :: Pokemon Hacken generate PokeCoins | Android/iOS', 1771, 1, 1468429599, 1468429599, 0, 0, 0, 0, 0, 0),
(26449, 26459, 'author', 'en', '▯{~{hackpack}~}▰ 13.07✥.2016 :: Code de Triche Pokemon free PokeCoins |. 2016', 1771, 1, 1468429599, 1468429599, 0, 0, 0, 0, 0, 0),
(26450, 26460, 'author', 'en', '▾{~HACKCHEA;TS~}▿ 13/07/2016 :: Pokemon hack jaibreak generate PokeCoins | 2016', 1771, 1, 1468429606, 1468429606, 0, 0, 0, 0, 0, 0),
(26451, 26461, 'author', 'en', '✤$fullhack✥ 13/07/2016 :: Pokemon Triche Gratuit generaTe PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468429610, 1468429610, 0, 0, 0, 0, 0, 0),
(26452, 26462, 'author', 'en', '©�♒�❼�;� :: Pokemon Glitch free PokeCoins, PokeBalls | Android', 1771, 1, 1468429612, 1468429612, 0, 0, 0, 0, 0, 0),
(26453, 26463, 'author', 'en', '﨏(~(❾�✢�❾)~)◀ :: Pokemon hack download generate PokeCoins | 2016', 1771, 1, 1468429621, 1468429621, 0, 0, 0, 0, 0, 0),
(26454, 26464, 'author', 'en', '♬|¯\\_❹❹❹_/¯|♭ :: Pokemon Ha\\ck 2016 ', 1771, 1, 1468429624, 1468429624, 0, 0, 0, 0, 0, 0),
(26455, 26465, 'author', 'en', '﨏(!(�.�❾❾)!) :: Pokemon Glitch and Glitches POkeBalls | AndrOi☇d/iOS', 1771, 1, 1468429627, 1468429627, 0, 0, 0, 0, 0, 0),
(26456, 26466, 'author', 'en', '◯(*(HACKEN.☡GINE)*) 13/07/2016 :: Pokemon APK IPA Mod hack PokeCoins', 1771, 1, 1468429631, 1468429631, 0, 0, 0, 0, 0, 0),
(26457, 26467, 'author', 'en', '▬HACK▭ ::. Pokemon cheats aPk PokeCoins | Android/iOS', 1771, 1, 1468429636, 1468429636, 0, 0, 0, 0, 0, 0),
(26458, 26468, 'author', 'en', '◨(~gethAck~)◨ :: Pokemon Hack Gratis ', 1771, 1, 1468429642, 1468429642, 0, 0, 0, 0, 0, 0),
(26459, 26469, 'author', 'en', '♫(!❻❻❻!) :: Pokemon cheats hacking free Pok♐eBalls | Android', 1771, 1, 1468429644, 1468429644, 0, 0, 0, 0, 0, 0),
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(26461, 26471, 'author', 'en', '♇!�/�/�/�/�/�/�/�/�/♈ :: Pokemon cheat review hack PokeCoins | 2016', 1771, 1, 1468429652, 1468429652, 0, 0, 0, 0, 0, 0),
(26462, 26472, 'author', 'en', '♗[!❺❺❺!]♘ :: Pokemon Tips and Tricks ', 1771, 1, 1468429656, 1468429656, 0, 0, 0, 0, 0, 0),
(26463, 26473, 'author', 'en', '❞((❾❾❾)) :: Pokemon hack android free PokeCoins, Pok*eBall✾s | Androi♹d/iOS', 1771, 1, 1468429669, 1468429669, 0, 0, 0, 0, 0, 0),
(26464, 26474, 'author', 'en', '♻{!{❸❸❸}!}♼ :: Pokemon Hack Cydia PokeBalls | Android/iOS', 1771, 1, 1468429686, 1468429686, 0, 0, 0, 0, 0, 0),
(26465, 26475, 'author', 'en', '☣{!{❻❻✯❻}!}☤ :: Pokemon T▦riche Gratuit ', 1771, 1, 1468429687, 1468429687, 0, 0, 0, 0, 0, 0),
(26466, 26476, 'author', 'en', '☆(!full.hack!) 13.07.2016 :: Pokemon cheat review Poke,CoIns, PokeBalls | 2016', 1771, 1, 1468429693, 1468429693, 0, 0, 0, 0, 0, 0),
(26467, 26477, 'author', 'en', '✣(!❸❸❸!)✤ :: Astuces Pour Pokemon free POkeC,o◩ins | iOS-Androi♤d', 1771, 1, 1468429696, 1468429696, 0, 0, 0, 0, 0, 0),
(26468, 26478, 'author', 'en', '♘_/¯❻❻❻¯\\_ :: Pokemon haCk free generate PokeBalls', 1771, 1, 1468429701, 1468429701, 0, 0, 0, 0, 0, 0),
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(26470, 26480, 'author', 'en', '◰{~❼❼❼~} :: Pokemon Juw♮elen Cheat hack POkeBalls.. | iOS-Android', 1771, 1, 1468429713, 1468429713, 0, 0, 0, 0, 0, 0),
(26471, 26481, 'author', 'en', '◾###Hackgenerator◾ 13:07:2016 :: Pokemon cheats for♠ ipad ', 1771, 1, 1468429716, 1468429716, 0, 0, 0, 0, 0, 0),
(26472, 26482, 'author', 'en', '✽(~(07/13/2016)~) :: Pokemon Hack Cheat Juwelen Deutsch! hack. PokeCoins |✴ iOS-Android', 1771, 1, 1468429726, 1468429726, 0, 0, 0, 0, 0, 0),
(26473, 26483, 'author', 'en', '✍¯\\_❸❸❸_/¯✂ :: Pokemon cheat/ sheet free PokeCoins, PokeBalls | Android', 1771, 1, 1468429733, 1468429733, 0, 0, 0, 0, 0, 0),
(26474, 26484, 'author', 'en', '☛[![ENGINE]!]☜ 13.07.2016 :: Pokemon cheat sheet PokeCoins | iOS-Android', 1771, 1, 1468429735, 1468429735, 0, 0, 0, 0, 0, 0),
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(26476, 26486, 'author', 'en', '❌###fullhack 13.07.2016 :: Pokemon cHeats online free PokeCoins, P.okeBAlls | iOS-Android', 1771, 1, 1468429738, 1468429738, 0, 0, 0, 0, 0, 0),
(26477, 26487, 'author', 'en', '☥❼❼�☛�☦ :: Pokemon hack iphone PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468429746, 1468429746, 0, 0, 0, 0, 0, 0),
(26478, 26488, 'author', 'en', '✡(*❻❻❻*) :: Pokemon Glitch generate PokeCoins | iOS-Android', 1771, 1, 1468429755, 1468429755, 0, 0, 0, 0, 0, 0),
(26479, 26489, 'author', 'en', '▬(~(❻❻❻)~)▭ :: Pokemon apk mega mod ', 1771, 1, 1468429756, 1468429756, 0, 0, 0, 0, 0, 0),
(26480, 26490, 'author', 'en', '✧❺❺❺✧ :: Pokemon cheats for. ipad free PokeCOins | 2016', 1771, 1, 1468429761, 1468429761, 0, 0, 0, 0, 0, 0),
(26481, 26491, 'author', 'en', '♮[!❶�;��❶!]♮ :: cheat Pokemon ha◛ck PokeCoins, PokeBalls | Android', 1771, 1, 1468429761, 1468429761, 0, 0, 0, 0, 0, 0),
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(26484, 26494, 'author', 'en', '△{~{cheat}~}▴ 13-07-2016 :..:.. Pokemon tips an❌d tricks PokeBalls | 2016', 1771, 1, 1468429780, 1468429780, 0, 0, 0, 0, 0, 0),
(26485, 26495, 'author', 'en', '◉[❼❼❼]◉ :: Pokemon hack work free PokeBalls', 1771, 1, 1468429782, 1468429782, 0, 0, 0, 0, 0, 0),
(26486, 26496, 'author', 'en', '◗~!~201\\6◘ 13:07:2016/ :: Pokemon OnLine Hack ', 1771, 1, 1468429783, 1468429783, 0, 0, 0, 0, 0, 0),
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(26488, 26498, 'author', 'en', '♌❻❻❻ :: Pokemon hack no download freE PokeCoins | 2016', 1771, 1, 1468429806, 1468429806, 0, 0, 0, 0, 0, 0),
(26490, 26500, 'author', 'en', '♷[!HACKPACK!]♸ 13.07.2016 :: Pokemon hack that works PokeBalls', 1771, 1, 1468429822, 1468429822, 0, 0, 0, 0, 0, 0),
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(26492, 26502, 'author', 'en', '☣HACKPACK☤ 13-07-2016 :: Pokemon hack iFile PokeBAlls | Android', 1771, 1, 1468429828, 1468429828, 0, 0, 0, 0, 0, 0),
(26493, 26503, 'author', 'en', '❁|_/¯generator¯\\_|❁ 13:07:2016 :: Pokemon Cheats Po✒keCoins, Poke◛Balls | Andro^id/iOS', 1771, 1, 1468429830, 1468429830, 0, 0, 0, 0, 0, 0),
(26494, 26504, 'author', 'en', '✸(*(❻❻❻)*) :: Pokemon cheat tool ^', 1771, 1, 1468429835, 1468429835, 0, 0, 0, 0, 0, 0),
(26495, 26505, 'author', 'en', '◳{{❼❼❼}}◴ :.:. Pokemon Generator, PokeCo♬ins, PokeBalls^ | 2016', 1771, 1, 1468429848, 1468429848, 0, 0, 0, 0, 0, 0),
(26496, 26506, 'author', 'en', '◝(FULLHACK)◞ 13/07/2016 :: Pokemon hack jaibre✽ak PokeCoins | Android/iOS', 1771, 1, 1468429850, 1468429850, 0, 0, 0, 0, 0, 0),
(26497, 26507, 'author', 'en', '☱[[❺❺❺]] :: Pokemon cheats ipad hack PokeBalls | Android', 1771, 1, 1468429851, 1468429851, 0, 0, 0, 0, 0, 0),
(26498, 26508, 'author', 'en', '◻#❽❽❽ :: Pokemon hacked apk free Poke/Coins', 1771, 1, 1468429859, 1468429859, 0, 0, 0, 0, 0, 0),
(26499, 26509, 'author', 'en', '◉[!hackpack!] :: Pokemon Hack 2016 hack PokeCoins▻, PokeBalls | iOS-Android', 1771, 1, 1468429870, 1468429870, 0, 0, 0, 0, 0, 0),
(26500, 26510, 'author', 'en', '◖(~13:07:2016~)◗ :: Pokemon ch✴eats androId PokeCoins |/ iOS-Android', 1771, 1, 1468429872, 1468429872, 0, 0, 0, 0, 0, 0),
(26501, 26511, 'author', 'en', '♄{*❷◠❷❷*} :: HOw to hack☮ Pokemon free PokeCoIns, PokeBalls | Androi!d/iOS', 1771, 1, 1468429874, 1468429874, 0, 0, 0, 0, 0, 0),
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(26503, 26513, 'author', 'en', '✴[HACK]✴ 13:07:2016 :◷: Pokemon cheat real PokeCoins | Android/iOS', 1771, 1, 1468429879, 1468429879, 0, 0, 0, 0, 0, 0),
(26504, 26514, 'author', 'en', '◬[~❸❸✒�☰��~]◬ :: Pokemon chea!ts free ', 1771, 1, 1468429892, 1468429892, 0, 0, 0, 0, 0, 0),
(26505, 26515, 'author', 'en', '✿{!{❺❺❺}!}❁ :: Pokemon Hack ifunbox free PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468429894, 1468429894, 0, 0, 0, 0, 0, 0),
(26506, 26516, 'author', 'en', '◕{*hackge◻nerato◜r*} 13.07.2016 :: how to get hack tool Pokemon free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468429895, 1468429895, 0, 0, 0, 0, 0, 0),
(26507, 26517, 'author', 'en', '◊(~(❺\\❺❺)~) :: Pokemon Cheat that works ', 1771, 1, 1468429901, 1468429901, 0, 0, 0, 0, 0, 0),
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(26509, 26519, 'author', 'en', '☔[![m❐egahack]!] 13/07/2016 :: Pokemon Hack Cheat Juwelen Deutsch! PoKeCoins | AndrOid/▮iOS', 1771, 1, 1468429913, 1468429913, 0, 0, 0, 0, 0, 0),
(26510, 26520, 'author', 'en', '◵[[❻^❻�❧��]]◶ :: Pokemon mod Pok\\e◂Coins |; Android', 1771, 1, 1468429919, 1468429919, 0, 0, 0, 0, 0, 0),
(26511, 26521, 'author', 'en', '!❷❷❷ :: Pokemon hack android no r|o|o|t| hack PokeCoins, PokeBalls | And.roid', 1771, 1, 1468429921, 1468429921, 0, 0, 0, 0, 0, 0),
(26512, 26522, 'author', 'en', '✱¯\\_❸❸❸_/¯✱ :: Pokemon h❉ack codes ', 1771, 1, 1468429923, 1468429923, 0, 0, 0, 0, 0, 0),
(26513, 26523, 'author', 'en', '✱|¯\\_❻❻❻_/¯| :: Pokemon cheats for iphone generate PokeCoins | iOS-AndroiD:', 1771, 1, 1468429924, 1468429924, 0, 0, 0, 0, 0, 0),
(26514, 26524, 'author', 'en', '✗{�✨��❺♈❺}✗ :: Pokemon cheat ios ', 1771, 1, 1468429945, 1468429945, 0, 0, 0, 0, 0, 0),
(26515, 26525, 'author', 'en', '▮{*{CHEATS}*} :: Pokemon cheat ios hack PokeBalls | 2,0,1,6,', 1771, 1, 1468429959, 1468429959, 0, 0, 0, 0, 0, 0),
(26516, 26526, 'author', 'en', '▹[HA✱CKEN*GINE]▹ 13-07-2016 :: Pokemon tips and tricks generate PokeCoins, PokeBalls | 2016', 1771, 1, 1468429966, 1468429966, 0, 0, 0, 0, 0, 0),
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(26518, 26528, 'author', 'en', '☑[![HACK.PACK]!] 13:07:2016 :: Pokemon hack tool f..r..e..e.. PokeBalls | AndRoid', 1771, 1, 1468429971, 1468429971, 0, 0, 0, 0, 0, 0),
(26519, 26529, 'author', 'en', '♶❻❻❻♶ :: Pokemon Hack 2016 genErate PokeCoins, Pok◞eBalls | 2016', 1771, 1, 1468429974, 1468429974, 0, 0, 0, 0, 0, 0),
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(26626, 26636, 'author', 'en', '☄(!❻❻❻!)☄ :: Pokemon on google play download  ', 1771, 1, 1468430608, 1468430608, 0, 0, 0, 0, 0, 0),
(26627, 26637, 'author', 'en', '[*❹❹.�▶��*] :: no survey hack goog,le pl,ay Pokemon  hack PokeBalls', 1771, 1, 1468430611, 1468430611, 0, 0, 0, 0, 0, 0),
(26628, 26638, 'author', 'en', '▪{*{❶❶❶}*}▫ :: Pokemon tricheUrs PokeCoins, PokeBalls', 1771, 1, 1468430614, 1468430614, 0, 0, 0, 0, 0, 0),
(26629, 26639, 'author', 'en', '☞hack☞ 13:07:2016 :: Pokemon Astuce ', 1771, 1, 1468430631, 1468430631, 0, 0, 0, 0, 0, 0),
(26630, 26640, 'author', 'en', '▸(!HACKENGINE!)▹ 13-07-2016 :: Pokemon cheats and tips haCk PokeBalls', 1771, 1, 1468430631, 1468430631, 0, 0, 0, 0, 0, 0),
(26631, 26641, 'author', 'en', '☐(13-07-20.16)☐ :: no survey hacK googLe play Pokemon  hack PokeC◃oins | Android/iOS', 1771, 1, 1468430633, 1468430633, 0, 0, 0, 0, 0, 0),
(26632, 26642, 'author', 'en', '◃{*❻❻❻*}◃ :.: Pokemon hack tool generate PokeCo,ins | iOS-Android', 1771, 1, 1468430636, 1468430636, 0, 0, 0, 0, 0, 0),
(26633, 26643, 'author', 'en', '♨(~(❽❽❽)~)♩ :: Pokemon Juwelen Hack gene.rate PokeCoins, PokeBalls | 201.6', 1771, 1, 1468430654, 1468430654, 0, 0, 0, 0, 0, 0),
(26634, 26644, 'author', 'en', '☞[*[❻❻❻▻]*]☟ :: Pokemon tips and tr♑i.cks ', 1771, 1, 1468430654, 1468430654, 0, 0, 0, 0, 0, 0),
(26635, 26645, 'author', 'en', '♇!mod♈ 13.07.2016 :: Pokemon tips for beginners ', 1771, 1, 1468430659, 1468430659, 0, 0, 0, 0, 0, 0),
(26636, 26646, 'author', 'en', '❎$❼❼❼❏ :: Pokemon Hack telecharger hack PokeCoiNs, PokeBalls | Android', 1771, 1, 1468430674, 1468430674, 0, 0, 0, 0, 0, 0),
(26637, 26647, 'author', 'en', '♉[HACKED]♉ :: Pokemon pir,ater telechaRger PokeCoins | 2016', 1771, 1, 1468430677, 1468430677, 0, 0, 0, 0, 0, 0),
(26638, 26648, 'author', 'en', '☿❸❸❸☀ :: Pokemon hack proof ', 1771, 1, 1468430678, 1468430678, 0, 0, 0, 0, 0, 0),
(26639, 26649, 'author', 'en', '♸[~❺❺❺~]♸ :: Pokemon hack that works free PokeCoins, PokeBalls | Android', 1771, 1, 1468430678, 1468430678, 0, 0, 0, 0, 0, 0),
(26640, 26650, 'author', 'en', '™❺❺❺™ :: Pokemon cheaT ios generate PokeCoins | Android', 1771, 1, 1468430681, 1468430681, 0, 0, 0, 0, 0, 0),
(26641, 26651, 'author', 'en', '♑((❾❾❾))♑ :: Pokemon hack for iphone free PokeC/oin\\s, P♋okeBalls | 2016', 1771, 1, 1468430714, 1468430714, 0, 0, 0, 0, 0, 0),
(26642, 26652, 'author', 'en', '﨏(~HackengiNe~)﨏 :: Pokemon hack no root PokeCoins', 1771, 1, 1468430717, 1468430717, 0, 0, 0, 0, 0, 0),
(26643, 26653, 'author', 'en', '✍[❸❸❸]✍ :: Pokemon ha☚ck ifunbox ', 1771, 1, 1468430717, 1468430717, 0, 0, 0, 0, 0, 0),
(26644, 26654, 'author', 'en', '❑❼❼❼❑ :: Pokemon G:litch ', 1771, 1, 1468430717, 1468430717, 0, 0, 0, 0, 0, 0),
(26645, 26655, 'author', 'en', '#LiVe>-@Partizani vs Ferencvaros l-ive St.re.am', 2073, 1, 1468430734, 1468430734, 0, 0, 0, 0, 0, 0),
(26646, 26656, 'author', 'en', '▢[*[❸❸❸]*]▢ :.:. Pokemon Hack Cheats Trainer generate PokeC♊oin♹s | Android', 1771, 1, 1468430739, 1468430739, 0, 0, 0, 0, 0, 0),
(26647, 26657, 'author', 'en', '♗{!❸❸❸!} :: Pokemon Ti☞ps and Tricks PokeBalls | Android', 1771, 1, 1468430742, 1468430742, 0, 0, 0, 0, 0, 0),
(26648, 26658, 'author', 'en', '◙(!❽❽❽!)◙ :: Pokemon cheats download free PokeCoins', 1771, 1, 1468430742, 1468430742, 0, 0, 0, 0, 0, 0),
(26649, 26659, 'author', 'en', '☗{~{GETHACK}~}☗ 13-07-201..6 :: Pokemon cheats onl\\ine hack PokeCoins | Android', 1771, 1, 1468430744, 1468430744, 0, 0, 0, 0, 0, 0),
(26650, 26660, 'author', 'en', '❌_/¯❹❹❹¯\\_❌ :: Pokemon hack mac ', 1771, 1, 1468430760, 1468430760, 0, 0, 0, 0, 0, 0),
(26651, 26661, 'author', 'en', '♀(*❸❸❸*)♁ :: Pokemon hack no survey  Hack PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468430764, 1468430764, 0, 0, 0, 0, 0, 0),
(26652, 26662, 'author', 'en', '★(!❾❾�✂��!)★ :: Pokemon apk cheats hac♓k PokeCoins, PokeBalls | Android', 1771, 1, 1468430765, 1468430765, 0, 0, 0, 0, 0, 0),
(26653, 26663, 'author', 'en', '☃{{❻❻❻}} :: Pokemon Cheats 2016 ', 1771, 1, 1468430768, 1468430768, 0, 0, 0, 0, 0, 0),
(26654, 26664, 'author', 'en', '♇fullhack♈ 13:07:2016 :: Astuces Pour Pokemon PokeCoins | iOS-Android', 1771, 1, 1468430769, 1468430769, 0, 0, 0, 0, 0, 0),
(26655, 26665, 'author', 'en', '☓hack☓ 13.07.2016 :: Pokemon cheat engine ', 1771, 1, 1468430781, 1468430781, 0, 0, 0, 0, 0, 0),
(26656, 26666, 'author', 'en', '♡{!{07.13.2016}!} :: Pokemon Tips and Tricks hack Pok♩eCoins, PokeBalls', 1771, 1, 1468430786, 1468430786, 0, 0, 0, 0, 0, 0),
(26657, 26667, 'author', 'en', '♡[*[❶❶❶]*] :: Pokemon cheats iphone hack PokeCoins | Android', 1771, 1, 1468430787, 1468430787, 0, 0, 0, 0, 0, 0),
(26658, 26668, 'author', 'en', '✖{{CHEAT}} 13:07:2*016 :: Pokemon cheats a▦pk free PokeCoins', 1771, 1, 1468430791, 1468430791, 0, 0, 0, 0, 0, 0),
(26659, 26669, 'author', 'en', '♟|_/¯CHEAT¯\\_|♟ 13:07:2016 :: Pokemon hack mod ', 1771, 1, 1468430811, 1468430811, 0, 0, 0, 0, 0, 0),
(26660, 26670, 'author', 'en', '♳fullhack♴ 13:07:2016 :: Pokemon cheat app gene✬rate PokeCoIns, PokeBalls | Android', 1771, 1, 1468430812, 1468430812, 0, 0, 0, 0, 0, 0),
(26661, 26671, 'author', 'en', '✦[!HACKTOOL!] 13-07-201.6 :: how to get hack tool Pokemon free PokeCoin/s', 1771, 1, 1468430818, 1468430818, 0, 0, 0, 0, 0, 0),
(26662, 26672, 'author', 'en', '◫(�!�!�!�!�!�!�!�!�!) :: how to hack Pokemon ', 1771, 1, 1468430830, 1468430830, 0, 0, 0, 0, 0, 0),
(26663, 26673, 'author', 'en', '♡!❽❽❽♢ :: Pokemon Hack Cydia hack PokeC..oin✷s, PokeBalls | 2016', 1771, 1, 1468430840, 1468430840, 0, 0, 0, 0, 0, 0),
(26664, 26674, 'author', 'en', '♥{~hack~} 13:07:.2016 :: how to hack Pokemon ', 1771, 1, 1468430844, 1468430844, 0, 0, 0, 0, 0, 0),
(26665, 26675, 'author', 'en', '◃(*(❹❹�♗�)*)◃ :: Pokemon cheat app free PokeCoins | 2016', 1771, 1, 1468430846, 1468430846, 0, 0, 0, 0, 0, 0),
(26666, 26676, 'author', 'en', '◡{~❾❾❾~} :: Pokemon cheats for! android generate PokeBalls', 1771, 1, 1468430846, 1468430846, 0, 0, 0, 0, 0, 0),
(26667, 26677, 'author', 'en', '◂#❼❼❼◃ :: Pokemon tips for beGinners generate PokeBalls* | iOS-Android', 1771, 1, 1468430858, 1468430858, 0, 0, 0, 0, 0, 0),
(26668, 26678, 'author', 'en', '✓{~{❶❶❶}~}✗ :: free g❇ame hack Tools Pokemon ', 1771, 1, 1468430865, 1468430865, 0, 0, 0, 0, 0, 0),
(26669, 26679, 'author', 'en', '♌(❶❶❶)♍ :: Pokemon cheats codes hack PokeBalls | 2016', 1771, 1, 1468430879, 1468430879, 0, 0, 0, 0, 0, 0),
(26670, 26680, 'author', 'en', '▪~!~En.gine 13.07.2016 :: Pokemon hack iph♍one no root ', 1771, 1, 1468430880, 1468430881, 0, 0, 0, 0, 0, 0),
(26671, 26681, 'author', 'en', '◡{!{�▾�❹❹}!} ::\\ Pokemon tiPs anD tricks free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468430885, 1468430885, 0, 0, 0, 0, 0, 0),
(26672, 26682, 'author', 'en', '✣(!(❻❻�◨��)!)✤ :: Pokemon cheats android◁ Poke♙Balls | Android/iOS', 1771, 1, 1468430893, 1468430893, 0, 0, 0, 0, 0, 0),
(26673, 26683, 'author', 'en', '☥{❹❹❹}☦ :: Pokemon Glitch free P:o:k:e:C:o:i:n:s: | Android/iOS', 1771, 1, 1468430902, 1468430902, 0, 0, 0, 0, 0, 0),
(26674, 26684, 'author', 'en', '☔{on♞line}☔ 13:07:2016 :: Pokemon tips ad.vanced ', 1771, 1, 1468430906, 1468430906, 0, 0, 0, 0, 0, 0),
(26675, 26685, 'author', 'en', '◎{!❼❼❼!}◎ :: Pokemon cheats for ipad PokeCo▾ins', 1771, 1, 1468430911, 1468430911, 0, 0, 0, 0, 0, 0),
(26676, 26686, 'author', 'en', '✠(!(❶❶❶)!)✡ :: Pokemon c:h:e:a:t:s: hacking PokeCoins | Android/iOS!', 1771, 1, 1468430915, 1468430915, 0, 0, 0, 0, 0, 0),
(26677, 26687, 'author', 'en', '☌|¯\\_hackengi|ne_/¯|☍ :: Pokemon c*heats for aNdroid free PokeCoiNs, PokeBalls', 1771, 1, 1468430924, 1468430924, 0, 0, 0, 0, 0, 0),
(26678, 26688, 'author', 'en', '☰¯\\_hacktool_/¯ 13☉-07-2016 :: Pokemon hack tool online ', 1771, 1, 1468430931, 1468430932, 0, 0, 0, 0, 0, 0),
(26679, 26689, 'author', 'en', '☣(!❺❺❺!) :: Pokemon apk mega Mod hack PokeBalls | Android/iOS☫', 1771, 1, 1468430951, 1468430951, 0, 0, 0, 0, 0, 0),
(26680, 26690, 'author', 'en', '◻¯\\_HACKENGINE_/¯◻ 13:07:2016 :: Pokemon hack no jailbreak fr☍ee PokECoins, PokeBalls | Android/iOS', 1771, 1, 1468430951, 1468430951, 0, 0, 0, 0, 0, 0),
(26681, 26691, 'author', 'en', '▿###megahack◠ 1✼3:07:2016 :: Pokemon Mod Apk ', 1771, 1, 1468430952, 1468430952, 0, 0, 0, 0, 0, 0),
(26682, 26692, 'author', 'en', '◃|_/¯�!��❾❾¯\\_|◃ :: Pokemon hack n*o* root free PokeBalls | 2016', 1771, 1, 1468430953, 1468430953, 0, 0, 0, 0, 0, 0),
(26683, 26693, 'author', 'en', '❘[~MOD~]❘ 13-07-2016 :: cheat Pokemon Po♚keCo.ins, Poke✨B.alls | iOS-AndroiD', 1771, 1, 1468430956, 1468430956, 0, 0, 0, 0, 0, 0),
(26684, 26694, 'author', 'en', '▰[!gethack!] 13-07-2016 :: Pokemon hack codes ', 1771, 1, 1468430978, 1468430978, 0, 0, 0, 0, 0, 0),
(26685, 26695, 'author', 'en', '♿$�.��❻❻♀ :: Pokemon hack apk hack PokeCoins, PokeBalls', 1771, 1, 1468430982, 1468430982, 0, 0, 0, 0, 0, 0),
(26686, 26696, 'author', 'en', '☁{*{MOD♐}*} 13-07-2016 :: Hack Para Pokemon ', 1771, 1, 1468431000, 1468431000, 0, 0, 0, 0, 0, 0),
(26687, 26697, 'author', 'en', '▥[![gethack]!] 13-07-2016 :: Pokemon Hack No Root hacK PokeCoins | 2016', 1771, 1, 1468431004, 1468431004, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(26689, 26699, 'author', 'en', '♵{!hacked!}♶ 13-07-..2016 :: Pokemon G;e;n;e;r;a;t;o;r; PokeBalls', 1771, 1, 1468431006, 1468431006, 0, 0, 0, 0, 0, 0),
(26690, 26700, 'author', 'en', '✼{*{july}*}✼ 13-07-2016 :: Pokemon hack ios free PokeBalls | Android', 1771, 1, 1468431029, 1468431029, 0, 0, 0, 0, 0, 0),
(26691, 26701, 'author', 'en', '◤[!❹❹❹!]◥ :: Pokemon cheats that acTually work hack PokeCoin.s | 2016', 1771, 1, 1468431033, 1468431033, 0, 0, 0, 0, 0, 0),
(26692, 26702, 'author', 'en', '◹{*�◼��❸❸*}◺ :: Pokemon Hack 2016 h✦ack PokeCoins, PokeBalls | 2016', 1771, 1, 1468431033, 1468431033, 0, 0, 0, 0, 0, 0),
(26693, 26703, 'author', 'en', '▸{!{HACKCHEATS}!}▸ 13.07.2016 :: Pokemon Generator ', 1771, 1, 1468431057, 1468431057, 0, 0, 0, 0, 0, 0),
(26694, 26704, 'author', 'en', '♔(*(�☳��❺❺)*)♕ :: Pokemon Cheats 2016 free PokeCoins', 1771, 1, 1468431059, 1468431059, 0, 0, 0, 0, 0, 0),
(26695, 26705, 'author', 'en', '☜(!(ONL♀INE)!) 13.07.2016 :: Code de Triche Pokemon hack PokeBalls | And▩roid/iOS', 1771, 1, 1468431060, 1468431060, 0, 0, 0, 0, 0, 0),
(26696, 26706, 'author', 'en', '♥[![HACK]!]♥ 13-07-201❍6 :: Pokemon apk cheats free PokeCoin,s,✲ PokeBalls | iOS-Android', 1771, 1, 1468431064, 1468431064, 0, 0, 0, 0, 0, 0),
(26697, 26707, 'author', 'en', '△{~{❺❺❺}~}△ :: Pokemon h.a.c.k. tool ', 1771, 1, 1468431083, 1468431083, 0, 0, 0, 0, 0, 0),
(26698, 26708, 'author', 'en', '▰!on♦line▱ 13-07-2016 :.: Pokemon hack android ', 1771, 1, 1468431085, 1468431085, 0, 0, 0, 0, 0, 0),
(26699, 26709, 'author', 'en', '▯(~❷�..��❷~) :: Pokemon hack for iphone PokeBalls | 2016', 1771, 1, 1468431088, 1468431088, 0, 0, 0, 0, 0, 0),
(26700, 26710, 'author', 'en', '✤(❷❷❷)✤ :: Pokemon hack codes generate PokeCoins, PokeBalls | iOS-And.roid', 1771, 1, 1468431090, 1468431090, 0, 0, 0, 0, 0, 0),
(26701, 26711, 'author', 'en', '▲|¯\\_❷❷❷_/¯|▲ :: Pokemon cheats and tips gener☜ate PokeCoins | 2016', 1771, 1, 1468431101, 1468431101, 0, 0, 0, 0, 0, 0),
(26702, 26712, 'author', 'en', '◧[~[❻❻❻◌]~]◧ :: Pokemon cheats reviews ', 1771, 1, 1468431104, 1468431104, 0, 0, 0, 0, 0, 0),
(26703, 26713, 'author', 'en', '◲ENGINE◲ 13:07:2016 :: Code de Triche Pokemon ', 1771, 1, 1468431108, 1468431108, 0, 0, 0, 0, 0, 0),
(26704, 26714, 'author', 'en', '✽(!en❎gine!)✽ 13:07:2016 :: Pokemon hack work ', 1771, 1, 1468431109, 1468431109, 0, 0, 0, 0, 0, 0),
(26705, 26715, 'author', 'en', '◧{*❻❻❻*}◨ :: Pokemon cheat sheet ', 1771, 1, 1468431115, 1468431115, 0, 0, 0, 0, 0, 0),
(26706, 26716, 'author', 'en', '☦~!~hackpack☦ 13.07.2016 :: Pokemon cheats real Po.keBalls♴ | 2016', 1771, 1, 1468431121, 1468431121, 0, 0, 0, 0, 0, 0),
(26707, 26717, 'author', 'en', '♏_/¯❸❸❸¯\\_♐ :: Pokemon Trucos ', 1771, 1, 1468431130, 1468431130, 0, 0, 0, 0, 0, 0),
(26708, 26718, 'author', 'en', '❃❼❼❼❅ :: Pokemon cheat codes ', 1771, 1, 1468431133, 1468431133, 0, 0, 0, 0, 0, 0),
(26709, 26719, 'author', 'en', '✵[!�.��❺*❺!] :|: Pokemon Hacker ', 1771, 1, 1468431147, 1468431147, 0, 0, 0, 0, 0, 0),
(26710, 26720, 'author', 'en', '❙{!�.��❺❺!}❚ :: Pokemon cheats online ', 1771, 1, 1468431153, 1468431153, 0, 0, 0, 0, 0, 0),
(26711, 26721, 'author', 'en', '♑|_/¯HAC✻KED¯\\_|♑ 13/07/2.016 :: Pokemon Tips and Tricks free PokeBalls | iOS-Android', 1771, 1, 1468431158, 1468431158, 0, 0, 0, 0, 0, 0),
(26712, 26722, 'author', 'en', '❊(!❺❺❺!)❋ :: Pokemon che.ats reviews ', 1771, 1, 1468431178, 1468431178, 0, 0, 0, 0, 0, 0),
(26713, 26723, 'author', 'en', '☜❷❷�.�� :: Pokemon hack ios ', 1771, 1, 1468431178, 1468431178, 0, 0, 0, 0, 0, 0),
(26714, 26724, 'author', 'en', '▩❽❽�☕��▪ :: Pokemon apk me..ga mod PokeBalls', 1771, 1, 1468431179, 1468431179, 0, 0, 0, 0, 0, 0),
(26715, 26725, 'author', 'en', '◩[[❶❶❶]]◪ :.: Pokemon hack no jailbreak ', 1771, 1, 1468431181, 1468431181, 0, 0, 0, 0, 0, 0),
(26716, 26726, 'author', 'en', '◸[*�^�^�^�^�^�^�^�^�^*]◸ :: Pokemon Hack No Survey ', 1771, 1, 1468431181, 1468431181, 0, 0, 0, 0, 0, 0),
(26717, 26727, 'author', 'en', '◊[*[❸�✸�❸▽]*] :: Pokemon Hack Cydia PokeCoins.', 1771, 1, 1468431202, 1468431202, 0, 0, 0, 0, 0, 0),
(26718, 26728, 'author', 'en', '☠|¯\\_CHEAT_/¯| 13:07:2016 :: Pokemon cheat ios ', 1771, 1, 1468431202, 1468431202, 0, 0, 0, 0, 0, 0),
(26719, 26729, 'author', 'en', '❙$MAY2016❙ :: Pokemon hack mac hack PokeCoins, PokeBalls | Android,/iOS', 1771, 1, 1468431226, 1468431226, 0, 0, 0, 0, 0, 0),
(26720, 26730, 'author', 'en', '▦(*(HACKGENERATOR)*) 13.07.2016 :: Pokemon hack ios hack PokeCoins, PoKeBalls |✴ 2016', 1771, 1, 1468431227, 1468431227, 0, 0, 0, 0, 0, 0),
(26721, 26731, 'author', 'en', '♺engine 13.07.2016 :: Pokemon Hacker free PokeCoins | iOS-Android', 1771, 1, 1468431231, 1468431231, 0, 0, 0, 0, 0, 0),
(26722, 26732, 'author', 'en', '❆|_/¯HACKENGINE¯\\_|❇ 13/07/2016 :: Pokemon hack jaibreak hack♽ PokeCoins, PokeBalls | i✺OS-.Andr.oid.', 1771, 1, 1468431249, 1468431249, 0, 0, 0, 0, 0, 0),
(26723, 26733, 'author', 'en', '♖{~❶❶❶~}♗ :: Pokemon hac;ked ap.k ', 1771, 1, 1468431253, 1468431253, 0, 0, 0, 0, 0, 0),
(26724, 26734, 'author', 'en', '❆❺❺❺❇ :: Pokemon hack i*p*h*o*n*e* generate PokeCoins, PokeBalls | 201◝6', 1771, 1, 1468431253, 1468431253, 0, 0, 0, 0, 0, 0),
(26725, 26735, 'author', 'en', '◼{~hackgenerator~} 13.07.2016 ::  free PokeBalls | Android/iOS.', 1771, 1, 1468431256, 1468431256, 0, 0, 0, 0, 0, 0),
(26726, 26736, 'author', 'en', '✲[*CHEAT*]✲ 13/07◢/2▴0❎16 :: Pokemon Trucos gEnerate Poke▶Coins, PokeBall^s | iOS-Android', 1771, 1, 1468431257, 1468431257, 0, 0, 0, 0, 0, 0),
(26727, 26737, 'author', 'en', '✶[~❷❷❷~]✷ :: Pokemon on google play do.w©nloAd  ', 1771, 1, 1468431276, 1468431276, 0, 0, 0, 0, 0, 0),
(26728, 26738, 'author', 'en', '♻~!~❻❻❻♻ :: Pokemon Hack No Roo.t hack P.o.k.e.B.a.l.l.s. | iOS-And❎roid', 1771, 1, 1468431281, 1468431281, 0, 0, 0, 0, 0, 0),
(26729, 26739, 'author', 'en', '◫[![ENG.INE]!]◬ 13.07.2016 :: Pokemon hack ifunbox freE PokeBalls | 2016', 1771, 1, 1468431281, 1468431281, 0, 0, 0, 0, 0, 0),
(26730, 26740, 'author', 'en', '♔{engine} 13-07-2016 :: cheat Pokemon ', 1771, 1, 1468431282, 1468431282, 0, 0, 0, 0, 0, 0),
(26731, 26741, 'author', 'en', '◳[!HACKTOOL!] 13-07-2016 :: Pokemon hack no download  Poke✥Coins, PokeBalls | Android', 1771, 1, 1468431303, 1468431303, 0, 0, 0, 0, 0, 0),
(26732, 26742, 'author', 'en', '❧~!~mod❧ 13:07:2016 :: Pokemon cheats real ', 1771, 1, 1468431308, 1468431308, 0, 0, 0, 0, 0, 0),
(26733, 26743, 'author', 'en', '♊((❺❺❺))♋ :: Pokemon cheat/s that actually work PokeCoins | Android/iOS', 1771, 1, 1468431309, 1468431309, 0, 0, 0, 0, 0, 0),
(26734, 26744, 'author', 'en', '♊[!❽❽❽!]♊ :: Pokemon hack android no root generate PokeCoins, PokeBalls◨ | 2016.', 1771, 1, 1468431310, 1468431310, 0, 0, 0, 0, 0, 0),
(26735, 26745, 'author', 'en', '❎{!{❶❶❶}!}❎ :◊: Pokemon Hack Tool APK IPA free PokeCoins | Android', 1771, 1, 1468431326, 1468431326, 0, 0, 0, 0, 0, 0),
(26736, 26746, 'author', 'en', '◼¯\\_07-13-2♜016_/¯◼ :: Pokemon hack safe PokeCoins | And☟roid/iOS', 1771, 1, 1468431330, 1468431330, 0, 0, 0, 0, 0, 0),
(26737, 26747, 'author', 'en', '◹~!~CHEAT 13.07.2016 :: Pokemon cheatS for iphone PokeBalls', 1771, 1, 1468431331, 1468431331, 0, 0, 0, 0, 0, 0),
(26738, 26748, 'author', 'en', '◡|¯\\_ONLINE_/¯|◡ 13:07:2016 :: Pokemon hack ipho♗ne free PokeCoi◃ns, PokeBalls | 2016', 1771, 1, 1468431335, 1468431335, 0, 0, 0, 0, 0, 0),
(26739, 26749, 'author', 'en', '◱###HACKGENERATOR:◱ 13/07/2016 :: Pokemon cheats download ', 1771, 1, 1468431338, 1468431338, 0, 0, 0, 0, 0, 0),
(26740, 26750, 'author', 'en', '☒(!(GENERATOR)!)☒ :: Telecharger Pokemon Hack Gratuit free PokeC✽oins, PokeB❙alls', 1771, 1, 1468431349, 1468431349, 0, 0, 0, 0, 0, 0),
(26741, 26751, 'author', 'en', '♌[!HACKP◪ACK!] :: Pokemon mo◗d Poke♢Balls', 1771, 1, 1468431352, 1468431352, 0, 0, 0, 0, 0, 0),
(26742, 26752, 'author', 'en', '☋(~(13.07.2016)~)☋ :: Pokemon APK IPA Mod ', 1771, 1, 1468431355, 1468431355, 0, 0, 0, 0, 0, 0),
(26743, 26753, 'author', 'en', '✵|_/¯13.07..20^1◂6^¯\\_|✵ :: Pokemon tri.cheurs ', 1771, 1, 1468431361, 1468431361, 0, 0, 0, 0, 0, 0),
(26744, 26754, 'author', 'en', '♶|¯\\_❸❸❸_/¯|♷ :: Pokemon i/pa hack hack PokeCoins', 1771, 1, 1468431370, 1468431370, 0, 0, 0, 0, 0, 0),
(26745, 26755, 'author', 'en', '☓{*hackgenerator*} 13/07/2016 :: Pokemon Cheats ', 1771, 1, 1468431377, 1468431377, 0, 0, 0, 0, 0, 0),
(26746, 26756, 'author', 'en', '◠[~❾�♔�❾~]◡ :: Pokemon hack free ', 1771, 1, 1468431387, 1468431387, 0, 0, 0, 0, 0, 0),
(26747, 26757, 'author', 'en', '☩(13:07:2016)☪ :: Pokemon cheat codes hac♥k PokeCoins | iOS-,Android', 1771, 1, 1468431393, 1468431393, 0, 0, 0, 0, 0, 0),
(26748, 26758, 'author', 'en', '❞❼❼❼❞ :: Pokemon hack no jaibreak free PokeCoins | iOS-Android', 1771, 1, 1468431400, 1468431400, 0, 0, 0, 0, 0, 0),
(26749, 26759, 'author', 'en', '♒{!❶❶❶!} :: Pokemon pirater telecharger generate PokeCoins☼ | ANdroid/i:OS', 1771, 1, 1468431402, 1468431402, 0, 0, 0, 0, 0, 0),
(26750, 26760, 'author', 'en', '❒[*❻❻♦❻*]❓ :: Pokemon tricheurs hack. Pok✓eCoins, PokeBAlls | iOS◹-Android', 1771, 1, 1468431409, 1468431409, 0, 0, 0, 0, 0, 0),
(26751, 26761, 'author', 'en', '▨(*(❶❶❶)*)▨ :: Pokemon APK IPA Mod ', 1771, 1, 1468431411, 1468431411, 0, 0, 0, 0, 0, 0),
(26752, 26762, 'author', 'en', '❈!❶❶❶❈ :: Pokemon hack ifunbox ', 1771, 1, 1468431424, 1468431424, 0, 0, 0, 0, 0, 0),
(26753, 26763, 'author', 'en', '▻(*❽❽❽*)▻ :: Pokemon cheat reAL ', 1771, 1, 1468431425, 1468431425, 0, 0, 0, 0, 0, 0),
(26754, 26764, 'author', 'en', '◴(~HACKGEN☏ERATOR~) 13-07-2016 :: free game hack tools Pokemon ', 1771, 1, 1468431434, 1468431434, 0, 0, 0, 0, 0, 0),
(26755, 26765, 'author', 'en', '♱(~(❽❽❽)~) :: Pokemon Onli..◩ne Hack free PokeCoIns, PokeBall.s', 1771, 1, 1468431434, 1468431434, 0, 0, 0, 0, 0, 0),
(26756, 26766, 'author', 'en', '☙((�❂�❽❽)) ::  ', 1771, 1, 1468431438, 1468431438, 0, 0, 0, 0, 0, 0),
(26757, 26767, 'author', 'en', '☫❹❄❹�☖��☬ :: Pokemon Hack Tool APK IPA ', 1771, 1, 1468431448, 1468431448, 0, 0, 0, 0, 0, 0),
(26758, 26768, 'author', 'en', '◵((13/07/.2016))◶ :: Pokemon hack android no root hack PokeCoins | iOS-AndroiD', 1771, 1, 1468431459, 1468431459, 0, 0, 0, 0, 0, 0),
(26759, 26769, 'author', 'en', '♴gethack :: Pokemon cheats for Mobile PokeCoins | A:n:d:r:o:i:d:', 1771, 1, 1468431460, 1468431460, 0, 0, 0, 0, 0, 0),
(26760, 26770, 'author', 'en', '✲(❾❾❾)✲ :: Pokemon mod PokeCoins | 2016', 1771, 1, 1468431463, 1468431463, 0, 0, 0, 0, 0, 0),
(26761, 26771, 'author', 'en', '❧[~❾❾�♍��~]★ :: Pokemon tips for n.oObs hack PokeCoins, PokeBalls | 2016', 1771, 1, 1468431472, 1468431472, 0, 0, 0, 0, 0, 0),
(26762, 26772, 'author', 'en', '▤{❼❼❼}▥ :: Pokemon hack tool PokeCoins', 1771, 1, 1468431474, 1468431474, 0, 0, 0, 0, 0, 0),
(26763, 26773, 'author', 'en', '☆[!❶❶❶!]✯ :: Pokemon cheAts for ipad ', 1771, 1, 1468431482, 1468431482, 0, 0, 0, 0, 0, 0),
(26764, 26774, 'author', 'en', '♚(~(❾❾❾)~)♛ :.:. Pokemon hack o☉Nline generate P▲okeCoins | 2016', 1771, 1, 1468431485, 1468431485, 0, 0, 0, 0, 0, 0),
(26765, 26775, 'author', 'en', '✈07-13-2016✈ :: Pokemon h☂acK◛ software hack PokeCo▵ins, Po✍keBalls | Android/iOS', 1771, 1, 1468431486, 1468431486, 0, 0, 0, 0, 0, 0),
(26766, 26776, 'author', 'en', '◌[!❽❽❽!]◌ :: Pokemon hack safe PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468431496, 1468431496, 0, 0, 0, 0, 0, 0),
(26767, 26777, 'author', 'en', '▰{!{❻�❌�❻}!}▰ :: Pokemon hack ifunbox generate PokeCoins | 2016', 1771, 1, 1468431498, 1468431498, 0, 0, 0, 0, 0, 0),
(26768, 26779, 'author', 'en', '❒[![❷❷❷]!] :: Pokemon ha☸C♃k i\\phone no root ', 1771, 1, 1468431510, 1468431510, 0, 0, 0, 0, 0, 0),
(26769, 26778, 'author', 'en', '♄(!(❺❺❺)!)♅ :☫: Pokemon Hack No Root ', 1771, 1, 1468431509, 1468431509, 0, 0, 0, 0, 0, 0),
(26770, 26780, 'author', 'en', '♠[~[MOD]~] 13-07-2016 :: Pokemon hack dOwnload hack PokeCoins, PokeBalls | iOS-.A☜ndroid', 1771, 1, 1468431521, 1468431521, 0, 0, 0, 0, 0, 0),
(26771, 26781, 'author', 'en', '❕((❽❽❽))❖ :: Pokemon Astuce hack PokeBalls | Android/iOS', 1771, 1, 1468431530, 1468431530, 0, 0, 0, 0, 0, 0),
(26772, 26782, 'author', 'en', '﨏|_/¯mod¯\\_|﨏 :: Pokemon cheat review ', 1771, 1, 1468431534, 1468431534, 0, 0, 0, 0, 0, 0),
(26773, 26783, 'author', 'en', '♮(!(❷❷❷)!)♮ :: Pokemon Hacker free PokeCoins | Andr☞oid,', 1771, 1, 1468431542, 1468431542, 0, 0, 0, 0, 0, 0),
(26774, 26784, 'author', 'en', '❂(*❸❸.�☺��*)❂ :: Pokemon hack activation code PokeCoins | An◽droid', 1771, 1, 1468431542, 1468431542, 0, 0, 0, 0, 0, 0),
(26775, 26785, 'author', 'en', '♠¯\\_engine_/¯♣ 13.07.2016 :: cheats for Pokemon generate PokeBalls | 2016', 1771, 1, 1468431552, 1468431552, 0, 0, 0, 0, 0, 0),
(26776, 26786, 'author', 'en', '◩(❽❽❽)◪ :: Pokemon mod hack PokeCoins', 1771, 1, 1468431556, 1468431556, 0, 0, 0, 0, 0, 0),
(26777, 26787, 'author', 'en', '▫¯\\_❶❶❶_/¯▬ :: Pokemon hack for io.s ', 1771, 1, 1468431565, 1468431565, 0, 0, 0, 0, 0, 0),
(26778, 26788, 'author', 'en', '❕[!❽❽❽!]❕ :: Pokemon tips tricks hack PokeCoins | Android', 1771, 1, 1468431567, 1468431567, 0, 0, 0, 0, 0, 0),
(26779, 26789, 'author', 'en', '✱{!{❼❼❼}!}✲ :✱: Pokemon tips advanced P,o,k,e,C,o,i,n,s,,, PokeBalls', 1771, 1, 1468431572, 1468431572, 0, 0, 0, 0, 0, 0),
(26780, 26790, 'author', 'en', '♠[!GENE▴RATOR!]♣ :: Pokemon cheat tool PokeCoiNs, PokeBalls | Android/iOS', 1771, 1, 1468431578, 1468431578, 0, 0, 0, 0, 0, 0),
(26781, 26791, 'author', 'en', '✠{*❺�☢��❺*}✠ :: Pokemon cheats and hacks hack PokeCoins | 2016', 1771, 1, 1468431580, 1468431580, 0, 0, 0, 0, 0, 0),
(26782, 26792, 'author', 'en', '☲$❻❻❻ :: Pokemon cheats fOr mobile generate PokeCoins | iOS-Android◃', 1771, 1, 1468431589, 1468431589, 0, 0, 0, 0, 0, 0),
(26783, 26793, 'author', 'en', '❔07/13/2016❔ :: cheats for Pokemon generate PokeCoins', 1771, 1, 1468431593, 1468431593, 0, 0, 0, 0, 0, 0),
(26784, 26794, 'author', 'en', '◺|_/¯❻❻❻¯\\_|◻ :: Pokemon hack no survey | ❙', 1771, 1, 1468431601, 1468431601, 0, 0, 0, 0, 0, 0),
(26785, 26795, 'author', 'en', '♳(!❻❻�◈�!)♳ :: Pokemon hack proof hack PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468431603, 1468431603, 0, 0, 0, 0, 0, 0),
(26786, 26796, 'author', 'en', '◧CHEA.T 13-07-2016 :: Pokemon Hacker gen.erate PokeCoins | 2016', 1771, 1, 1468431614, 1468431614, 0, 0, 0, 0, 0, 0),
(26787, 26797, 'author', 'en', '❙{{❹�☟�❹}} :: Pokemon apk cheats g..e..n..e..r..a..t..e.. PokeBalls', 1771, 1, 1468431635, 1468431635, 0, 0, 0, 0, 0, 0),
(26788, 26798, 'author', 'en', '❖{*{❼♑❼❼}*}❗ :: Pokemon mod ', 1771, 1, 1468431636, 1468431636, 0, 0, 0, 0, 0, 0),
(26789, 26799, 'author', 'en', '☖(!❺❺❺!)☗ :: Telecharger Pokemon Hack Gratuit ', 1771, 1, 1468431636, 1468431636, 0, 0, 0, 0, 0, 0),
(26790, 26800, 'author', 'en', '▦(!(❸❸❸)!) ::. Pokemon cheats reViews free PokeC|oins | Android☩', 1771, 1, 1468431637, 1468431637, 0, 0, 0, 0, 0, 0),
(26791, 26801, 'author', 'en', '♺[!mod!]♺ :: c..h..e..a..t.. Pokemon hack PokeCo.ins, PokeBalls', 1771, 1, 1468431638, 1468431638, 0, 0, 0, 0, 0, 0),
(26792, 26802, 'author', 'en', '☊[*❽❽❽*]☋ :: Pokemon Cheats 2016 generate PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468431660, 1468431660, 0, 0, 0, 0, 0, 0),
(26793, 26803, 'author', 'en', '▤❷❷❷ :: Pokemon hack cheats hack PokeCoins | 2016', 1771, 1, 1468431662, 1468431662, 0, 0, 0, 0, 0, 0),
(26794, 26804, 'author', 'en', '☇[~HACKCHEATS~] :: Pokemon cheat codes PokeCoins | Android/iOS', 1771, 1, 1468431662, 1468431662, 0, 0, 0, 0, 0, 0),
(26795, 26805, 'author', 'en', '▻((13-07-20◔16)) :: Pokemon Hack No Root PokeCoins', 1771, 1, 1468431663, 1468431663, 0, 0, 0, 0, 0, 0),
(26796, 26806, 'author', 'en', '✥|¯\\_❻❻❻_/¯|✥ :/:/ Code de Triche Pokemon ', 1771, 1, 1468431687, 1468431687, 0, 0, 0, 0, 0, 0),
(26797, 26807, 'author', 'en', '▸[~Meg!ahack~]▸ :: Pokemon Pirater generate PokeBalls | An☳DroI✭d/iOS', 1771, 1, 1468431687, 1468431687, 0, 0, 0, 0, 0, 0),
(26798, 26808, 'author', 'en', '❁¯\\_❶❶❶_/¯ :: Pokemon hack t◖ool onlin♤e free PokeCoins, PokeBalls | Android', 1771, 1, 1468431692, 1468431692, 0, 0, 0, 0, 0, 0),
(26799, 26809, 'author', 'en', '©{!1✈3..07.2016!}® :: Pokemon hack android n.o. root PokeBalls | Android/iOS', 1771, 1, 1468431710, 1468431710, 0, 0, 0, 0, 0, 0),
(26800, 26810, 'author', 'en', '☢{~❾�❇�❾~}☣ :: Pokemon Hack free Po!keCoins, PokeBalls', 1771, 1, 1468431712, 1468431712, 0, 0, 0, 0, 0, 0),
(26801, 26811, 'author', 'en', '✵[❺❺❺] :: Pokemon cheats hacking free PokeBalls | 2016', 1771, 1, 1468431714, 1468431714, 0, 0, 0, 0, 0, 0),
(26802, 26812, 'author', 'en', '♵(*❷❷❷*)♵ :: Pokemon Onlin☠e Generator generate PokeCoins, PokeBalls | Android', 1771, 1, 1468431717, 1468431717, 0, 0, 0, 0, 0, 0),
(26803, 26813, 'author', 'en', '◭[*[07-13-2☛016]*]◭ :: Pokemon Hack Cydia ', 1771, 1, 1468431737, 1468431737, 0, 0, 0, 0, 0, 0),
(26804, 26814, 'author', 'en', '◘❶❶❶.◙ :: Pokemon hack mod hack POkeC✫o♥ins | iOS-Android', 1771, 1, 1468431738, 1468431738, 0, 0, 0, 0, 0, 0),
(26805, 26815, 'author', 'en', '❜[*cheat*]❜ 13.\\07.2016 :: Pokemon hack mac PokeCoins, Pok▯eB♔alls', 1771, 1, 1468431739, 1468431739, 0, 0, 0, 0, 0, 0),
(26806, 26816, 'author', 'en', '▥~!~❾❾❾▦ :: Pokemon Trucos hack Poke◷Balls | 2016', 1771, 1, 1468431740, 1468431740, 0, 0, 0, 0, 0, 0),
(26807, 26817, 'author', 'en', '❑{*{❽❽❽}*} :,:, Pokemon hack no jailbreak ', 1771, 1, 1468431761, 1468431761, 0, 0, 0, 0, 0, 0),
(26808, 26818, 'author', 'en', '[TV-HD+.*] Ludogorets vs Mladost Podgorica l.i.v.e S.t.r.e.a.m 2016', 2050, 1, 1468431778, 1468431778, 0, 0, 0, 0, 0, 0),
(26809, 26819, 'author', 'en', '✠❺❺�◂�✠ :: Pokemon cheat th^at works ', 1771, 1, 1468431781, 1468431781, 0, 0, 0, 0, 0, 0),
(26810, 26820, 'author', 'en', '♹###M^O^D^ 13.07.2016 :: Pokemon Hack No Survey free PokeCoins | Android', 1771, 1, 1468431782, 1468431782, 0, 0, 0, 0, 0, 0),
(26811, 26821, 'author', 'en', '✹{HACKGEN♗ERAT.OR}✺ 13/07/2016 :: Pokemon hack mod ', 1771, 1, 1468431783, 1468431783, 0, 0, 0, 0, 0, 0),
(26812, 26822, 'author', 'en', '✤((july.2016))✤ :: Pokemon h,ack iphone no root◎ generate PokeBalls | 2016', 1771, 1, 1468431783, 1468431783, 0, 0, 0, 0, 0, 0),
(26813, 26823, 'author', 'en', '♍|_/¯❼❼❼¯\\_| :: Pokemon Hack iPhone iPad Android ', 1771, 1, 1468431783, 1468431783, 0, 0, 0, 0, 0, 0),
(26814, 26824, 'author', 'en', '♏CHEATS♏ :: Pokemon Cheats online PokeBalls | Android/iOS', 1771, 1, 1468431806, 1468431806, 0, 0, 0, 0, 0, 0),
(26815, 26825, 'author', 'en', '❎((ME.GAHACK)) :: Pokemon cheats for ipho☑ne ', 1771, 1, 1468431807, 1468431807, 0, 0, 0, 0, 0, 0),
(26816, 26826, 'author', 'en', '✉(*❸❸❸*) :: Pokemon Hack ', 1771, 1, 1468431811, 1468431811, 0, 0, 0, 0, 0, 0),
(26817, 26827, 'author', 'en', '✿❾❾❾✿ :: Pokemon tips tricks free PokeBalls | Android', 1771, 1, 1468431811, 1468431811, 0, 0, 0, 0, 0, 0),
(26818, 26828, 'author', 'en', '❃❺❺❺ :: Pokemon hack mac ', 1771, 1, 1468431813, 1468431813, 0, 0, 0, 0, 0, 0),
(26819, 26829, 'author', 'en', '♹~!~CHEAT♹ :: Pokemon Cheats 2016 ge➤nerate PokeC◰oins', 1771, 1, 1468431830, 1468431830, 0, 0, 0, 0, 0, 0),
(26820, 26830, 'author', 'en', '▮[❸❸❸] :: Pokemon hack tool g◬enerat✢or ', 1771, 1, 1468431830, 1468431830, 0, 0, 0, 0, 0, 0),
(26821, 26831, 'author', 'en', '#❼❼❼ :: Pokemon tips for beginners PokeBalls | i*O*S*-*A*n*d*r*o*i*d*', 1771, 1, 1468431836, 1468431836, 0, 0, 0, 0, 0, 0),
(26822, 26832, 'author', 'en', '☙[!2016!] 13-.07-2016 :: Pokemon cheats ipad generate♆ PokeCoins | 2016', 1771, 1, 1468431838, 1468431838, 0, 0, 0, 0, 0, 0),
(26823, 26833, 'author', 'en', '☻{!❻❻☹❻!}☼ :: Pokemon cheats aNd hacks PokeCoins | iOS-◽AnDroid', 1771, 1, 1468431855, 1468431855, 0, 0, 0, 0, 0, 0),
(26824, 26834, 'author', 'en', '◑|_/¯HACKENGINE¯\\_| :: Pokemon hack fo;r andr◩oid PokeCoins, PokeBalls | Android', 1771, 1, 1468431859, 1468431859, 0, 0, 0, 0, 0, 0),
(26825, 26835, 'author', 'en', '◮(*(❶❶❶)*)◮ :: how to hack Pokemon ', 1771, 1, 1468431859, 1468431859, 0, 0, 0, 0, 0, 0),
(26826, 26836, 'author', 'en', '▵((❾❾❾))▶ :: Pokemon hack Tool ', 1771, 1, 1468431883, 1468431883, 0, 0, 0, 0, 0, 0),
(26827, 26837, 'author', 'en', '▢{{❽❽❽}}▢ :: Pokemon hack online ', 1771, 1, 1468431887, 1468431887, 0, 0, 0, 0, 0, 0),
(26828, 26838, 'author', 'en', '▿((�◭��❽❽)) :: Pokemon cheat engine hack PokeCoins | 2016', 1771, 1, 1468431898, 1468431898, 0, 0, 0, 0, 0, 0),
(26829, 26839, 'author', 'en', '☢{!gethack!}☣ ::◮ Pokemon chea..ts download PokeCoins | Android/iOS', 1771, 1, 1468431906, 1468431906, 0, 0, 0, 0, 0, 0),
(26830, 26840, 'author', 'en', '♰❷❷❷♱ :: Pokemon H.acken free PokeBalls | Android', 1771, 1, 1468431908, 1468431908, 0, 0, 0, 0, 0, 0),
(26831, 26841, 'author', 'en', '◘{!{❺❺❺}!} :: Pokemon tips cheats ', 1771, 1, 1468431922, 1468431922, 0, 0, 0, 0, 0, 0),
(26832, 26842, 'author', 'en', '◓{!❼❼❼!}◓ :: Pokemon Triche Gratuit PokeCoins, PokeBalls', 1771, 1, 1468431925, 1468431925, 0, 0, 0, 0, 0, 0),
(26833, 26843, 'author', 'en', '❔!❺❺�◑�❕ :: Pokemon tips for beginners free PokeCoins | iOS-Android', 1771, 1, 1468431925, 1468431925, 0, 0, 0, 0, 0, 0),
(26834, 26844, 'author', 'en', '➤|_/¯❾❾❾¯\\_|™ :: Pokemon Hack C.h.e.a.t.s. Trainer generate PokeCoins, PokeBalls | AndroId', 1771, 1, 1468431933, 1468431933, 0, 0, 0, 0, 0, 0),
(26835, 26845, 'author', 'en', '♪{*{hackgeneraTor}*}♪ :: Pokemon cheaTs android ', 1771, 1, 1468431936, 1468431936, 0, 0, 0, 0, 0, 0),
(26836, 26846, 'author', 'en', '♂{{❼❼❼}} :: Pokemon cheats hacking free PokeCoins, PokeBalls', 1771, 1, 1468431950, 1468431950, 0, 0, 0, 0, 0, 0),
(26837, 26847, 'author', 'en', '♥¯\\_❷❷❷_/¯ :: Pokemon Hack generate PokeCoins | iOS-Android', 1771, 1, 1468431951, 1468431951, 0, 0, 0, 0, 0, 0),
(26838, 26848, 'author', 'en', '▽|¯\\_❼�✨��.�,�_/¯|▽ :: Pokemon HAck Gratis hack POkeCoi☵ns, PokeBalls', 1771, 1, 1468431951, 1468431951, 0, 0, 0, 0, 0, 0),
(26839, 26849, 'author', 'en', '◚!❸❸❸◛ :: Pokemon cheats ios free PokeBalls | Android', 1771, 1, 1468431960, 1468431960, 0, 0, 0, 0, 0, 0),
(26840, 26850, 'author', 'en', '◪¯\\_CHEAT_/¯◫ 13-07-2016 :: Pokemon hack tool download PokeCoIns, PokeBalls | Androi❏d/iOS', 1771, 1, 1468431965, 1468431965, 0, 0, 0, 0, 0, 0),
(26841, 26851, 'author', 'en', '◊[~❽❽,❽~] :\\:\\ Pokemon Hack 2016 free PokeBalls | Android/iOS', 1771, 1, 1468431977, 1468431977, 0, 0, 0, 0, 0, 0),
(26842, 26852, 'author', 'en', '❜2016❝ :: Pokemon hack iphOne no root hack PokeCoins | Android/iOS', 1771, 1, 1468431980, 1468431980, 0, 0, 0, 0, 0, 0),
(26843, 26853, 'author', 'en', '◣[[HA.CKED]]◣ 13-07-2016 :: Pokemon cheat tool☄ free PokeCoins, PokeBalls | iOS-ANdroid', 1771, 1, 1468431982, 1468431982, 0, 0, 0, 0, 0, 0),
(26844, 26854, 'author', 'en', '☓(*❽❽�♂�*)☔ :: Pokemon cheats that actuall☘y work hack PokeCoins, PokEBalls | 2016', 1771, 1, 1468431988, 1468431988, 0, 0, 0, 0, 0, 0),
(26845, 26855, 'author', 'en', '♬¯\\_HACKGENERATOR_/¯♭ 13-△07-2016 :: Pokemon hack no survey  PokeCoins | 2016', 1771, 1, 1468431995, 1468431995, 0, 0, 0, 0, 0, 0),
(26846, 26856, 'author', 'en', '◐{!{CHEAT}!}◐ 13:07:2016 :: AsTuces Pour Pokemon ', 1771, 1, 1468432002, 1468432002, 0, 0, 0, 0, 0, 0),
(26847, 26857, 'author', 'en', '▼(~(hAck)~) :: Pokemon hack ifunbox hack PokeBalls', 1771, 1, 1468432008, 1468432008, 0, 0, 0, 0, 0, 0),
(26848, 26858, 'author', 'en', '◗(❽❽❽)◘ :: Pokemon Hac✿ken genera❔t♂e PokeBalls | 2016', 1771, 1, 1468432029, 1468432029, 0, 0, 0, 0, 0, 0),
(26849, 26859, 'author', 'en', '❅❾❾❾❅ :: Pokemon Hacken h\\a\\c\\k\\ PokeBalls | iOS-Android', 1771, 1, 1468432030, 1468432030, 0, 0, 0, 0, 0, 0),
(26850, 26860, 'author', 'en', '◭[~cheats~]◭ 13/07/2016 :: Pokemon hack app generate PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468432034, 1468432034, 0, 0, 0, 0, 0, 0),
(26851, 26861, 'author', 'en', '☊(*(07-13-2016)*) :: Pokemon apk mega mod PokeBalls | 2016', 1771, 1, 1468432042, 1468432042, 0, 0, 0, 0, 0, 0),
(26852, 26862, 'author', 'en', '☷((❻❻❻)) :: Pokemon mod PokeBalls | Android/iO▾S', 1771, 1, 1468432058, 1468432058, 0, 0, 0, 0, 0, 0),
(26853, 26863, 'author', 'en', '✵[[❺❺❺]]✵ :: cheats for Pokemon free PokeCoins', 1771, 1, 1468432058, 1468432058, 0, 0, 0, 0, 0, 0),
(26854, 26864, 'author', 'en', '☑{engine}☑ 13/07/2016 ::. Pokemon Glitch and Glitches ', 1771, 1, 1468432071, 1468432071, 0, 0, 0, 0, 0, 0),
(26855, 26865, 'author', 'en', '♯ENGINE♰ 13-07-2016 :: Pokemon che\\at code entry generate POkeBalls | Android', 1771, 1, 1468432082, 1468432082, 0, 0, 0, 0, 0, 0),
(26856, 26866, 'author', 'en', '☂(*(❸❸❸)*) :: Pokemon Trucos hack PokeCoins*', 1771, 1, 1468432085, 1468432085, 0, 0, 0, 0, 0, 0),
(26857, 26867, 'author', 'en', '◻generato*r 13:07:2016 :: Pokemon ch♌eats hackin♨g ', 1771, 1, 1468432088, 1468432088, 0, 0, 0, 0, 0, 0),
(26858, 26868, 'author', 'en', '☸(~(❷❷❷)~)☹ :: Pokemon hack No d☉ownload  PokeBalls', 1771, 1, 1468432088, 1468432088, 0, 0, 0, 0, 0, 0),
(26859, 26869, 'author', 'en', '◕(!(❼❼❼)!)◖ :: Pokemon cheats reviews generate PokeCoins, PokeBalls | A♮Ndroid', 1771, 1, 1468432097, 1468432097, 0, 0, 0, 0, 0, 0),
(26860, 26870, 'author', 'en', '◀_/¯❻❻❻¯\\_◀ :: Pokemon hack ifunbox ', 1771, 1, 1468432109, 1468432109, 0, 0, 0, 0, 0, 0),
(26861, 26871, 'author', 'en', '♴{*❼♸❼❼*}♴ :: Pokemon cheats and tips h.ack PokeCoins | Android/iOS', 1771, 1, 1468432111, 1468432111, 0, 0, 0, 0, 0, 0),
(26862, 26872, 'author', 'en', '★{!{hackgenerAtor}!}☆ 13-07-2016 :: Hack Par✉a Pokemon free PokeCOins | Android/iOS', 1771, 1, 1468432114, 1468432114, 0, 0, 0, 0, 0, 0),
(26863, 26873, 'author', 'en', '☕[![07◸/13/2016]!]☖ :: Pokemon cheats hacks free P☔okeBalls | 2016', 1771, 1, 1468432115, 1468432115, 0, 0, 0, 0, 0, 0),
(26864, 26874, 'author', 'en', '◞((❺❺❺))◟ :: Pokemon pir.a♟te.r te✬lecharger generate PokeBalls | 2016', 1771, 1, 1468432125, 1468432125, 0, 0, 0, 0, 0, 0),
(26865, 26875, 'author', 'en', '❄HA♆CK.CHEATS 13/0.7/2016 :: Pokemon cheats that actually work PokeCoins, PokeBalls | 2!0!1!6!', 1771, 1, 1468432134, 1468432134, 0, 0, 0, 0, 0, 0),
(26866, 26876, 'author', 'en', '☐[~ONLINE~] 13/07/2016 :: Pokemon haCk for android PokeCoins | iOS-Android', 1771, 1, 1468432136, 1468432136, 0, 0, 0, 0, 0, 0),
(26867, 26877, 'author', 'en', '☫|_/¯HACK¯\\_|☫ :: Pokemon tips and che□at☠s hack Po^keCoins, PokeBalls | 2016', 1771, 1, 1468432140, 1468432140, 0, 0, 0, 0, 0, 0),
(26868, 26878, 'author', 'en', '▪[!MAY.!] 13..07.2016 :: Pokemon hack no survey  ', 1771, 1, 1468432141, 1468432141, 0, 0, 0, 0, 0, 0),
(26869, 26879, 'author', 'en', '[!❼❼❼▵!] :: Pokemon cheats hacks fr☶ee PokeCoins, PokeBa◄lls', 1771, 1, 1468432150, 1468432150, 0, 0, 0, 0, 0, 0),
(26870, 26880, 'author', 'en', '✦###HACKTOOL✧ :: Pokemon H.a.c.k. 2016 PokeCoins, PokeBalls | 2016', 1771, 1, 1468432155, 1468432155, 0, 0, 0, 0, 0, 0),
(26871, 26881, 'author', 'en', '◶|¯\\_13.07.2016_/¯|◷ :: Pokemon cheat real generate Poke.Coins, PokeBalls', 1771, 1, 1468432157, 1468432157, 0, 0, 0, 0, 0, 0),
(26872, 26882, 'author', 'en', '☑[❾❾❾]☒ :: Pokemon ha,ck app ', 1771, 1, 1468432167, 1468432167, 0, 0, 0, 0, 0, 0),
(26873, 26883, 'author', 'en', '☏{*{cheats}*}☏ 13.07.2016 :: Pokemon cheat That works PokeCoins | Android*/iO.S', 1771, 1, 1468432183, 1468432183, 0, 0, 0, 0, 0, 0),
(26874, 26884, 'author', 'en', '❐(!(❹❹❹)!)❑ :: Pokemon chea;ts reviews ', 1771, 1, 1468432183, 1468432183, 0, 0, 0, 0, 0, 0),
(26875, 26885, 'author', 'en', '✂(~❻❻❻~)✒ :: Pokemon hacK safe ', 1771, 1, 1468432187, 1468432187, 0, 0, 0, 0, 0, 0),
(26876, 26886, 'author', 'en', '◠{*❾❾❾*}◡ :: Pokemon hac:k❅ apk ', 1771, 1, 1468432199, 1468432199, 0, 0, 0, 0, 0, 0),
(26877, 26887, 'author', 'en', '♞$❻❻❻♟ :: Pokemon hack ipad hack PokeCoins | And;roid', 1771, 1, 1468432213, 1468432213, 0, 0, 0, 0, 0, 0),
(26878, 26888, 'author', 'en', '▷#❹❹❹▷ :: free g.a.m.e. hack tool!s Pokemon PokeCoins | iOS-Android◥', 1771, 1, 1468432216, 1468432216, 0, 0, 0, 0, 0, 0),
(26879, 26889, 'author', 'en', '♟❺❺❺ :: Pokemon Cheat✵s 2016 ', 1771, 1, 1468432224, 1468432224, 0, 0, 0, 0, 0, 0),
(26880, 26890, 'author', 'en', '❔enGine❕ 13.07..2!016 :: Pokemon Generator generate PokeBalls |. 2016', 1771, 1, 1468432227, 1468432227, 0, 0, 0, 0, 0, 0),
(26881, 26891, 'author', 'en', '◕~!~2016◕ 13/07/2.016 :: Pokemon h!ack apk PokeCoi▢ns, PokeBalls | Android', 1771, 1, 1468432232, 1468432232, 0, 0, 0, 0, 0, 0),
(26882, 26892, 'author', 'en', '☭{~{HACKGENERATOR}~}☮ 13/07►/♩2*016 :: Pokemon cheats real generAte PokeCoins', 1771, 1, 1468432238, 1468432238, 0, 0, 0, 0, 0, 0),
(26883, 26893, 'author', 'en', '♮[![o.nline]!]♯ 13.07.2016 :: Pokemon hack online P❌okeBalls | iOS-AndR▽oid', 1771, 1, 1468432239, 1468432239, 0, 0, 0, 0, 0, 0),
(26884, 26894, 'author', 'en', '♝[~❾❾❾~]♞ :/:/ Pokemon hack mod apk. generate PokeCoins, PokeBalls | 2016', 1771, 1, 1468432246, 1468432246, 0, 0, 0, 0, 0, 0),
(26885, 26895, 'author', 'en', '◈hackpack◉ 13:07:2016 :: Pokemon cheats download ☧', 1771, 1, 1468432249, 1468432249, 0, 0, 0, 0, 0, 0),
(26886, 26896, 'author', 'en', '❌[[❾❾❾]] :: Pokemon cheats and tips g,e,n,e,r,a,t,e, PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468432255, 1468432255, 0, 0, 0, 0, 0, 0),
(26887, 26897, 'author', 'en', '▷{!{hackengine}!} 13.07.2016 :: Pokemon Juwelen Hack ', 1771, 1, 1468432261, 1468432261, 0, 0, 0, 0, 0, 0),
(26888, 26898, 'author', 'en', '■❸❸�❆�□ :: Pokemon hack mod apk ', 1771, 1, 1468432262, 1468432262, 0, 0, 0, 0, 0, 0),
(26889, 26899, 'author', 'en', '◆{!❻❻❻!} :: Pokemon cheat thAt works PokeCoins | 2016', 1771, 1, 1468432269, 1468432269, 0, 0, 0, 0, 0, 0),
(26890, 26900, 'author', 'en', '➤{{07.13..2016}} :: Pokemon Juwele✰n Chea.t generAte PokeC^oins | Android', 1771, 1, 1468432272, 1468432272, 0, 0, 0, 0, 0, 0),
(26891, 26901, 'author', 'en', '✧ENGINE✨ 13.07.2016 :: Pokemon hack for ios ', 1771, 1, 1468432276, 1468432276, 0, 0, 0, 0, 0, 0),
(26892, 26902, 'author', 'en', '▰[CHEATS]▰ 13-07-2016 :: Pokemon hack telecharger hack PokeCoins', 1771, 1, 1468432283, 1468432283, 0, 0, 0, 0, 0, 0),
(26893, 26903, 'author', 'en', '✰{~{❺❺�*��}~}✰ :: Pokemon Cheats 201,6 ', 1771, 1, 1468432288, 1468432288, 0, 0, 0, 0, 0, 0),
(26894, 26904, 'author', 'en', '✤{~{❺❺❺}~} :: Pokemon cheat codes hack PokeBalls | Android', 1771, 1, 1468432295, 1468432295, 0, 0, 0, 0, 0, 0),
(26895, 26905, 'author', 'en', '✽¯\\_❽❽❽_/¯✾ :: Pokemon Hack Chea®t J.uw✦elen DEutsch! free Poke,Coins,☰ PokeBalls | iOS-Android', 1771, 1, 1468432295, 1468432295, 0, 0, 0, 0, 0, 0),
(26896, 26906, 'author', 'en', '❗(*(❸❸❸)*) :: Pokemon che.ats anD tips ', 1771, 1, 1468432298, 1468432298, 0, 0, 0, 0, 0, 0),
(26897, 26907, 'author', 'en', '➤[*[generator]*]™ 13-07-20/16 :: Pokemon apk cheats free PokeCoins | 2016', 1771, 1, 1468432311, 1468432311, 0, 0, 0, 0, 0, 0),
(26898, 26908, 'author', 'en', '☄[*MEGAHACK*]☄ 13-07-2016 ::  PokeCoins, PokeBalls | Android', 1771, 1, 1468432318, 1468432318, 0, 0, 0, 0, 0, 0),
(26899, 26909, 'author', 'en', '☵{{hacked}}☶ 13-07-2016 :: Pokemon tricheurs ', 1771, 1, 1468432323, 1468432323, 0, 0, 0, 0, 0, 0),
(26900, 26910, 'author', 'en', '⚓❻❻❻ :: Pokemon cheat t✧hat works free PokeCoins, P◷okeBalls | 2016', 1771, 1, 1468432327, 1468432327, 0, 0, 0, 0, 0, 0),
(26901, 26911, 'author', 'en', '☴(!g.ethack!)☵ :: Pokemon on google play◻ download  free PokeCoins, PokeBalls | 2016', 1771, 1, 1468432343, 1468432343, 0, 0, 0, 0, 0, 0),
(26902, 26912, 'author', 'en', '◓{❽❽❽}◓ :: Pokemon hack No root free PokeCoins | Android', 1771, 1, 1468432348, 1468432348, 0, 0, 0, 0, 0, 0),
(26903, 26913, 'author', 'en', '◰(!(❽❽�|��)!) :: Pokemon Cheats free generate PokeBalls | Android', 1771, 1, 1468432358, 1468432358, 0, 0, 0, 0, 0, 0),
(26904, 26914, 'author', 'en', '☀_/¯07:13:2016¯\\_ :: Pokemon hack that works PokeBalls | A!ndroid/iOS', 1771, 1, 1468432359, 1468432359, 0, 0, 0, 0, 0, 0),
(26905, 26915, 'author', 'en', '☻(~(cheats)~) 13:07:2016 :: Pokemon cheat that works PokeBalls', 1771, 1, 1468432363, 1468432363, 0, 0, 0, 0, 0, 0),
(26906, 26916, 'author', 'en', '♔{{❺❺❺}}♔ :: Pokemon hack work ', 1771, 1, 1468432365, 1468432365, 0, 0, 0, 0, 0, 0),
(26907, 26917, 'author', 'en', '◫|_/¯❹❹❹¯\\_|◬ :: Pokemon Tric.he ', 1771, 1, 1468432369, 1468432369, 0, 0, 0, 0, 0, 0),
(26908, 26918, 'author', 'en', '©[~[❸❸❸]~]® :: Pokemon hack for iphone free P..okeCoi▢ns, PokeBalls | Android/iOS', 1771, 1, 1468432379, 1468432379, 0, 0, 0, 0, 0, 0),
(26909, 26919, 'author', 'en', '♑�,�,�,�,�,�,�,�,�,♑ :: Pokemon H❂ack 2016 ', 1771, 1, 1468432381, 1468432381, 0, 0, 0, 0, 0, 0),
(26910, 26920, 'author', 'en', '◀(!(�◫�❽❽)!) :: Pokemon cheats ipad g/en.erate PokeCoins, PokeBalls | Android', 1771, 1, 1468432384, 1468432384, 0, 0, 0, 0, 0, 0),
(26911, 26921, 'author', 'en', '✱{~{❹❹❹}~}✱ :: Pokemon Hack Cheats Trainer free PokeCoins, PokeBalls | 2016', 1771, 1, 1468432389, 1468432389, 0, 0, 0, 0, 0, 0),
(26912, 26922, 'author', 'en', '▯{!{❻❻❻}!}▯ :: Pokemon ha.c◚K work ', 1771, 1, 1468432391, 1468432391, 0, 0, 0, 0, 0, 0),
(26913, 26923, 'author', 'en', '☊((❻❻❻))☋ :: Pokemon Hack No Root ', 1771, 1, 1468432402, 1468432402, 0, 0, 0, 0, 0, 0),
(26914, 26924, 'author', 'en', '☽{!❻�.�☾�❻!}☾ :: Pokemon Cheats 2016 PokeCoins | 2016', 1771, 1, 1468432405, 1468432405, 0, 0, 0, 0, 0, 0),
(26915, 26925, 'author', 'en', '❙[!❼❼❼!]❙ :: Pokemon Pirater telecharger ', 1771, 1, 1468432406, 1468432406, 0, 0, 0, 0, 0, 0),
(26916, 26926, 'author', 'en', '♌(❷❷❷) :: Pokemon hack for android ', 1771, 1, 1468432411, 1468432411, 0, 0, 0, 0, 0, 0),
(26917, 26927, 'author', 'en', '☨¯\\_getha.ck_/¯ 13/07/2016 :: Pokemon tIps advanced PokeCoins | And♣roid', 1771, 1, 1468432414, 1468432414, 0, 0, 0, 0, 0, 0),
(26918, 26928, 'author', 'en', '✿(*(mod)*)✿ 13.07.2016 :: Pokemon cheats codes h.a.c.k. PokeCoins,^ PokeBalls | Android/iOS', 1771, 1, 1468432424, 1468432424, 0, 0, 0, 0, 0, 0),
(26919, 26929, 'author', 'en', '♘❸❸❸ :: Pokemon hack if©unbox hack PokeCoins', 1771, 1, 1468432427, 1468432427, 0, 0, 0, 0, 0, 0),
(26920, 26930, 'author', 'en', '◓❷❷❷ :: Pokemon hack tool generate PokeCoins | iOS.-Android', 1771, 1, 1468432428, 1468432428, 0, 0, 0, 0, 0, 0),
(26921, 26931, 'author', 'en', '✲[![❾❾✡❾]!]✳ :: Pokemon hack no survey  generate PokeCoIns, PokeBalls | 2016', 1771, 1, 1468432438, 1468432438, 0, 0, 0, 0, 0, 0),
(26922, 26932, 'author', 'en', '◻[[MAY]]◼ :: Pokemon cheats for ip☽ad generate PokeCoi.ns, PokeBalls', 1771, 1, 1468432445, 1468432445, 0, 0, 0, 0, 0, 0),
(26923, 26933, 'author', 'en', '☩¯\\_HACKENGINE_/¯☪ 13/07/2016 :: Pokemon cheats a❌pk PokeBalls | AndroId', 1771, 1, 1468432448, 1468432448, 0, 0, 0, 0, 0, 0),
(26924, 26934, 'author', 'en', '☐(CHEATS)☑ 13.07.2016 :: Pokemon cheats and hacks ', 1771, 1, 1468432450, 1468432450, 0, 0, 0, 0, 0, 0),
(26925, 26935, 'author', 'en', '☼[[❻❻❻]]☽ :✱: Pokemon hack androi..♞d no root hack PokeBalls | Android/iOS', 1771, 1, 1468432457, 1468432457, 0, 0, 0, 0, 0, 0),
(26926, 26936, 'author', 'en', '☬(~❽❽❽~)☭ :: Pokemon cheat code entry free Po✬keCoins, PokeBalls', 1771, 1, 1468432480, 1468432480, 0, 0, 0, 0, 0, 0),
(26927, 26937, 'author', 'en', '♪[~❶❶❶~] :: Pokemon mod ', 1771, 1, 1468432487, 1468432487, 0, 0, 0, 0, 0, 0),
(26928, 26938, 'author', 'en', '❀[!❾❾❾!] :: Pokemon hack toOl free. PokeCoins | iOS-Android', 1771, 1, 1468432490, 1468432490, 0, 0, 0, 0, 0, 0),
(26929, 26939, 'author', 'en', '✧{*�☗�❷❷*} :: Pokemon Hack No Root generate PokeBalls | Android/iOS', 1771, 1, 1468432492, 1468432492, 0, 0, 0, 0, 0, 0),
(26930, 26940, 'author', 'en', '◅❼❼❼◅ :: Pokemon hack ifile hack PokeBalls | iOS-Android', 1771, 1, 1468432501, 1468432501, 0, 0, 0, 0, 0, 0),
(26931, 26941, 'author', 'en', '♘(~❸❸❸~)♙ :: Pokemon hack no jailbreak ', 1771, 1, 1468432506, 1468432506, 0, 0, 0, 0, 0, 0),
(26932, 26942, 'author', 'en', '✣❸❸❸✤ :: Telecharger Pokemon Hack Gr▽at™uit PokeBa❍lls', 1771, 1, 1468432509, 1468432509, 0, 0, 0, 0, 0, 0),
(26933, 26943, 'author', 'en', '◙[~[❷❷:❷]~] :: Pokemon Juwelen Ha.ck PoKeCoins | Android', 1771, 1, 1468432512, 1468432512, 0, 0, 0, 0, 0, 0),
(26934, 26944, 'author', 'en', '☪c☗heats☪ 13/07/20☀16 ::; Pokemon H✿a♀ck Gratis hack PokeCoins | Android/iOS', 1771, 1, 1468432513, 1468432513, 0, 0, 0, 0, 0, 0),
(26935, 26945, 'author', 'en', '☂(!❼❼❼!)☂ :.:. Pokemon cheat review hack PokeCoins | iOS-Android', 1771, 1, 1468432523, 1468432523, 0, 0, 0, 0, 0, 0),
(26936, 26946, 'author', 'en', '❔(!(fullhack)!)❔ 13/07/2016 :: no survey hack google play Pokemon  hack PokeCoins | Android/iOS', 1771, 1, 1468432529, 1468432529, 0, 0, 0, 0, 0, 0),
(26937, 26947, 'author', 'en', '♾(*(GETHACK)*) 13:07:2016 :: Pokemon hack Apk geneRate PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468432530, 1468432530, 0, 0, 0, 0, 0, 0),
(26938, 26948, 'author', 'en', '♌(!(2016)!) :: Pokemon hack ifunbox generate PokeCoins', 1771, 1, 1468432533, 1468432533, 0, 0, 0, 0, 0, 0),
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(26940, 26950, 'author', 'en', '♶{*{❷❷❷}*} :: Pokemon cheats hacking PokeBAlls', 1771, 1, 1468432542, 1468432542, 0, 0, 0, 0, 0, 0),
(26941, 26951, 'author', 'en', '❀(!❾❾❾!)❃ :: Pokemon cheats that actu..ally Work ', 1771, 1, 1468432552, 1468432552, 0, 0, 0, 0, 0, 0),
(26942, 26952, 'author', 'en', '♎[*[�,�,�,�,�,�,�,�,�,]*]♎ :\\: Pokemon Ju.welen Cheat ', 1771, 1, 1468432552, 1468432552, 0, 0, 0, 0, 0, 0),
(26943, 26953, 'author', 'en', '♅❷❷❷♆ :: Pokemon tips cheats hack PokeCoins | iOS-◀An.droiD', 1771, 1, 1468432553, 1468432553, 0, 0, 0, 0, 0, 0),
(26944, 26954, 'author', 'en', '❌[![HACK..TOOL]!]❍ 13-07-2016 :: Pokemon APK IPA Mod PokeCoins, PokE:Balls | iOS-Android', 1771, 1, 1468432556, 1468432556, 0, 0, 0, 0, 0, 0),
(26945, 26955, 'author', 'en', '✴{hacked} 13.07.2016 :: Pokemon hack downl.oad f^ree PokeCoins, PokeBalls | 2016', 1771, 1, 1468432574, 1468432574, 0, 0, 0, 0, 0, 0),
(26946, 26956, 'author', 'en', '○$❽❽❽ :: Pokemon hack for Ios ', 1771, 1, 1468432574, 1468432574, 0, 0, 0, 0, 0, 0),
(26947, 26957, 'author', 'en', '❙_/¯H;A;C;K;E;D;¯\\_ :: Pokemon hack no survey  generate PokeBalls | iOS-Android', 1771, 1, 1468432581, 1468432581, 0, 0, 0, 0, 0, 0),
(26948, 26958, 'author', 'en', '♃[!❷❷☥❷!]♄ :: Pokemon hack android ', 1771, 1, 1468432581, 1468432581, 0, 0, 0, 0, 0, 0),
(26949, 26959, 'author', 'en', '★!2016 13-07-2016 :: Pokemon Trucos ,', 1771, 1, 1468432591, 1468432591, 0, 0, 0, 0, 0, 0),
(26950, 26960, 'author', 'en', '◟(*❹❹❹*)◟ :: Pokemon Juwelen Ch☣eat ', 1771, 1, 1468432605, 1468432605, 0, 0, 0, 0, 0, 0),
(26951, 26961, 'author', 'en', '♗[~[❸.❸❸]~]♗ :: Pokemon Triche Gratuit generate PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468432615, 1468432615, 0, 0, 0, 0, 0, 0),
(26952, 26962, 'author', 'en', '☢[[HACKPACK◙]] :: Pokemon cheats hacking ', 1771, 1, 1468432623, 1468432623, 0, 0, 0, 0, 0, 0),
(26953, 26963, 'author', 'en', '✿{!{❸�,��❸}!} :: Pokemon hack that works PokeCoin♮s | 2016', 1771, 1, 1468432623, 1468432623, 0, 0, 0, 0, 0, 0),
(26954, 26964, 'author', 'en', '☭(*(❹❹❹)*)☮ ::  PokeCoins, PokeBalls | And▤roid', 1771, 1, 1468432626, 1468432626, 0, 0, 0, 0, 0, 0),
(26955, 26965, 'author', 'en', '◔[!�✪�❽❽▸!]◕ :: Pokemon hack no download PokeBalls | Android/iOS', 1771, 1, 1468432640, 1468432640, 0, 0, 0, 0, 0, 0),
(26956, 26966, 'author', 'en', '◽!❻❻❻ :: Pokemon hack for android generate PokeCoins, PokeBall.s', 1771, 1, 1468432648, 1468432648, 0, 0, 0, 0, 0, 0),
(26957, 26967, 'author', 'en', '☮|_/¯onli..ne¯\\_|☰ 13.07.2016 :: Pokemon cheats apk PoKe!Balls | Android', 1771, 1, 1468432649, 1468432649, 0, 0, 0, 0, 0, 0),
(26958, 26968, 'author', 'en', '❂[!❽❽❽!] :: Pokemon Hack 2016 free PokeCoins, PokeBall▹s♵ | iOS.-Android', 1771, 1, 1468432651, 1468432651, 0, 0, 0, 0, 0, 0),
(26959, 26969, 'author', 'en', '♴[❷❷❷]♵ :: Pokemon Hack hack PokeCoins, P;okeBalls | iOS-Android', 1771, 1, 1468432658, 1468432658, 0, 0, 0, 0, 0, 0),
(26960, 26970, 'author', 'en', '☌[!13.07.2016!]☌ :: free game hack tools Pokemon hack PokeBalls', 1771, 1, 1468432663, 1468432663, 0, 0, 0, 0, 0, 0),
(26961, 26971, 'author', 'en', '◰❻❻❻◰ ::  generate PokeCoins, PokeBalls', 1771, 1, 1468432674, 1468432674, 0, 0, 0, 0, 0, 0),
(26962, 26972, 'author', 'en', '✽###2016✽ :: Pokemon Hacken PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468432674, 1468432674, 0, 0, 0, 0, 0, 0),
(26963, 26973, 'author', 'en', '❉[*❽❽❽*] :: Pokemon hack no download ▰ hack PokeCoins,◌ PokeBAlls | iOS-Android', 1771, 1, 1468432675, 1468432675, 0, 0, 0, 0, 0, 0),
(26964, 26974, 'author', 'en', '■{july}□ :.:. Pokemon hack no download ', 1771, 1, 1468432681, 1468432681, 0, 0, 0, 0, 0, 0),
(26965, 26975, 'author', 'en', '❏{!{❸❸❸}!} :: Pokemon c;heats hacks ..', 1771, 1, 1468432687, 1468432687, 0, 0, 0, 0, 0, 0),
(26966, 26976, 'author', 'en', '◃❶❶❶◄ :: Pokemon hack jaibreak hAck PokeCoins | iOS-Android', 1771, 1, 1468432699, 1468432699, 0, 0, 0, 0, 0, 0),
(26967, 26977, 'author', 'en', '♲ON◨LINE♲ 13/07/2016 :: Pokemon hack android☮ No root ', 1771, 1, 1468432701, 1468432701, 0, 0, 0, 0, 0, 0),
(26968, 26978, 'author', 'en', '☋[~[MEGAHACK]~] 13/07/2016 :: Pokemon ipa hack ', 1771, 1, 1468432701, 1468432701, 0, 0, 0, 0, 0, 0),
(26969, 26979, 'author', 'en', '﨏{MOD}﨏 13.07.2016 :: Pokemon hack codes Pok◒eCoins,☘ PokeBalls', 1771, 1, 1468432704, 1468432704, 0, 0, 0, 0, 0, 0),
(26970, 26980, 'author', 'en', '◩❼❼❼◩ :: Pokemon cheats tOol hack PokeBalls | Android', 1771, 1, 1468432720, 1468432720, 0, 0, 0, 0, 0, 0),
(26971, 26981, 'author', 'en', '☤[!❼❼�▫�!]☥ :: Pokemon hack work ', 1771, 1, 1468432723, 1468432723, 0, 0, 0, 0, 0, 0),
(26972, 26982, 'author', 'en', '☆[~[2016]~] 13:07:2016 :: Pokemon Astuce free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468432726, 1468432726, 0, 0, 0, 0, 0, 0),
(26973, 26983, 'author', 'en', '▨{~{HACKENGINE}~}▩ 13:07:2016 :: Pokemon hAck and cheats hack, PokeCoins | A.n.d.r.o.i.d./.i.O.S.', 1771, 1, 1468432729, 1468432729, 0, 0, 0, 0, 0, 0),
(26974, 26984, 'author', 'en', '▢|_/¯❺❺❺¯\\_| :: Pokemon tricheurs ', 1771, 1, 1468432737, 1468432737, 0, 0, 0, 0, 0, 0),
(26975, 26985, 'author', 'en', '❚{{HACKPACK}}❛ 13.07.2016 :: Pokemon cheats for mobile free Poke*Coins | Android', 1771, 1, 1468432751, 1468432751, 0, 0, 0, 0, 0, 0),
(26976, 26986, 'author', 'en', '▭{!{HA✽CKED}!}▭ 13.07.▬2016 :: Pokemon Hack 2016 hack PokeBalls | 2016', 1771, 1, 1468432760, 1468432760, 0, 0, 0, 0, 0, 0),
(26977, 26987, 'author', 'en', '♔❺❺❺♕ :: Pokemon cheats codes ', 1771, 1, 1468432765, 1468432765, 0, 0, 0, 0, 0, 0),
(26978, 26988, 'author', 'en', '☃{*{CHEATS}*}☄ 13.07.2016 :: Pokemon h,ack ifile POkeBalls | Android', 1771, 1, 1468432775, 1468432775, 0, 0, 0, 0, 0, 0),
(26979, 26989, 'author', 'en', '♏❸❸❸ :: Pokemon cheat review ', 1771, 1, 1468432776, 1468432776, 0, 0, 0, 0, 0, 0),
(26980, 26990, 'author', 'en', '☍{~{❾❾�♕��}~} :: Pokemon O▿nl♒ine Hack generate⚓ PokeCoins | iOS✤-Andro☄i☠d', 1771, 1, 1468432783, 1468432783, 0, 0, 0, 0, 0, 0),
(26981, 26991, 'author', 'en', '○❻❻❻○ :: Pokemon hack iphone ', 1771, 1, 1468432795, 1468432795, 0, 0, 0, 0, 0, 0),
(26982, 26992, 'author', 'en', '◹❷❷❷ :: Pokemon hack^ and cheats generate PokeBalls | Android/iOS', 1771, 1, 1468432806, 1468432806, 0, 0, 0, 0, 0, 0),
(26983, 26993, 'author', 'en', '♔{!{❸❸❸}!}♔ :: Pokemon cheats for mobile ', 1771, 1, 1468432807, 1468432807, 0, 0, 0, 0, 0, 0),
(26984, 26994, 'author', 'en', '♍_/¯hackcheats¯\\_♍ 13-07-2016 :: Pokemon c♕heat engine ', 1771, 1, 1468432809, 1468432809, 0, 0, 0, 0, 0, 0),
(26985, 26995, 'author', 'en', '►{*{�\\�❺❺}*}► :: Pokemon Cheats 2016 ', 1771, 1, 1468432809, 1468432809, 0, 0, 0, 0, 0, 0),
(26986, 26996, 'author', 'en', '✮((❾❾❾))✰ :: Pokemon tricheurs hack PokeCoinS | iOS-Andr^oid', 1771, 1, 1468432818, 1468432818, 0, 0, 0, 0, 0, 0),
(26987, 26997, 'author', 'en', '☂{~{❾❾❾}~}☃ :: Pokemon cheat code entry f⚓ree PokeCoins', 1771, 1, 1468432830, 1468432830, 0, 0, 0, 0, 0, 0),
(26988, 26998, 'author', 'en', '✵(~❶❶❶~)✶ :: how to get hack☨ tool Pokemon gEnerate PokeBalls', 1771, 1, 1468432834, 1468432834, 0, 0, 0, 0, 0, 0),
(26989, 26999, 'author', 'en', '♖(*(❽❽❽)*)♗ :: Pokemon hack apk f,r,e,e, PokeBalls | 2016', 1771, 1, 1468432834, 1468432834, 0, 0, 0, 0, 0, 0),
(26990, 27000, 'author', 'en', '♐(!(cheat)!)♐ 13-07-2016 :: Pokemon cheats apk PokeCoins, PokeBalls | Android', 1771, 1, 1468432835, 1468432835, 0, 0, 0, 0, 0, 0),
(26991, 27001, 'author', 'en', '☶❻❻❻☷ :: Pokemon hack for android hack PokeBalls | An.droid/iOS.', 1771, 1, 1468432839, 1468432839, 0, 0, 0, 0, 0, 0),
(26992, 27002, 'author', 'en', '[[.*.EsPn^TV**..]] Fleetwood Town vs Liverpool 2016 L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468432841, 1468433041, 0, 0, 0, 0, 0, 0),
(26993, 27003, 'author', 'en', '✧❾❾❾✧ :: Pokemon hack Telechar☁ger hack PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468432852, 1468432852, 0, 0, 0, 0, 0, 0),
(26994, 27004, 'author', 'en', '❼❼❼ :: Pokemon hack✠ maC PokeCoins, PokeBalls | Android/iOS', 1771, 1, 1468432857, 1468432857, 0, 0, 0, 0, 0, 0),
(26995, 27005, 'author', 'en', '❘~!~hackgenerator 13.07.2016 :: Pokemon Hacken PokeBalls | Android/iOS', 1771, 1, 1468432857, 1468432857, 0, 0, 0, 0, 0, 0),
(26996, 27006, 'author', 'en', '☏{*{generator}*} 13.07.2016 :: Pokemon tips cheats hack PokeBalls | 2016', 1771, 1, 1468432864, 1468432864, 0, 0, 0, 0, 0, 0),
(26997, 27007, 'author', 'en', '✣[megahack] :: Telecharger Pokemon Hack Gratuit ', 1771, 1, 1468432873, 1468432873, 0, 0, 0, 0, 0, 0),
(26998, 27008, 'author', 'en', '◳{!HACKGENERATOR!}◴ 13:07:2016 :: how to get hack t.o.o.l. Pokemon ', 1771, 1, 1468432880, 1468432880, 0, 0, 0, 0, 0, 0),
(26999, 27009, 'author', 'en', '◑❻❻❻◒ :: Pokemon hac▨k ifunbox generate PokeCoins, PokeBAlls | Android', 1771, 1, 1468432880, 1468432880, 0, 0, 0, 0, 0, 0),
(27000, 27010, 'author', 'en', '☙|_/¯❼❼❼¯\\_|☙ :: Pokemon Apk◗ cheats ', 1771, 1, 1468432883, 1468432883, 0, 0, 0, 0, 0, 0),
(27001, 27011, 'author', 'en', '☔¯\\_❷❷❷_/¯☕ :: Pokemon hack tool gene;rator hacK PokeBalls | 2016', 1771, 1, 1468432888, 1468432888, 0, 0, 0, 0, 0, 0),
(27002, 27012, 'author', 'en', '♶((hac◦kTool)) 13:07:20☵16 :: Pokemon t▬ips tricks free PokeBalls', 1771, 1, 1468432909, 1468432909, 0, 0, 0, 0, 0, 0),
(27003, 27013, 'author', 'en', '❄{~engine~} 13-07-2☳016 :: Pokemon cheat/s hacks free PokeCoins, PokeBalls | IOS-Android', 1771, 1, 1468432925, 1468432925, 0, 0, 0, 0, 0, 0),
(27004, 27014, 'author', 'en', '♉{!{❾❾❾}!}♊ :: how to hack Pokemon free PokeCoins, PokeBalls | 2016', 1771, 1, 1468432930, 1468432930, 0, 0, 0, 0, 0, 0),
(27005, 27015, 'author', 'en', '✥{~❸�◮��❸~}✦ :: free game hack tools Pokemon ,', 1771, 1, 1468432934, 1468432934, 0, 0, 0, 0, 0, 0),
(27006, 27016, 'author', 'en', '✫[❺❺�◬�]✬ :: Pokemon hack jaibr;eak ', 1771, 1, 1468432937, 1468432937, 0, 0, 0, 0, 0, 0),
(27007, 27017, 'author', 'en', '✰[*❶❶❶*]✰ :: Pokemon Hacken hack Pok☬eCoins, PokeBalls | Android', 1771, 1, 1468432939, 1468432939, 0, 0, 0, 0, 0, 0),
(27008, 27018, 'author', 'en', '▪((HAC,KENGINE))▫ :^:^ Pokemon Glitch h❕aCk PokeBalls | 2016', 1771, 1, 1468432950, 1468432950, 0, 0, 0, 0, 0, 0),
(27009, 27019, 'author', 'en', '♾❼❼❼ :: Pokemon cheats android▷ free PokeCoins', 1771, 1, 1468432956, 1468432956, 0, 0, 0, 0, 0, 0),
(27010, 27020, 'author', 'en', '❚{~hac^kt,ool~} 13.07.2016✶ :: Pokemon cheat real h♵ack PokeBalls | iOS-Android', 1771, 1, 1468432960, 1468432960, 0, 0, 0, 0, 0, 0),
(27011, 27021, 'author', 'en', '◽[[❾❾❾]] :: Pokemon ha◤ck activa♊tion code g◵enerate PokeBalls | Android/iOS', 1771, 1, 1468432962, 1468432962, 0, 0, 0, 0, 0, 0),
(27012, 27022, 'author', 'en', '♲{*{megAhack}*}♳ 13:07:❜2016 :: Pokemon on google play download  ', 1771, 1, 1468432965, 1468432965, 0, 0, 0, 0, 0, 0),
(27013, 27023, 'author', 'en', '©|_/¯hack¯\\_| 13/07/2016 :: cheat Pokemon hack P\\o\\k\\e\\C\\o\\i\\n\\s\\,\\ PoKeBalls | 2016', 1771, 1, 1468432974, 1468432974, 0, 0, 0, 0, 0, 0);
INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(27014, 27024, 'author', 'en', '♗(~GENERATOR~)♘ 13.07.2016 :: Pokemon HAck No Root ge:nerate PokeCoins, PokeB.alls\\ | An:droid', 1771, 1, 1468432977, 1468432977, 0, 0, 0, 0, 0, 0),
(27015, 27025, 'author', 'en', '▮[!GENERATOR!]▮ 13/07/2016 :: Pokemon Mod Apk free PokeCoins', 1771, 1, 1468432982, 1468432982, 0, 0, 0, 0, 0, 0),
(27016, 27026, 'author', 'en', '☄¯\\_❼❼❼_/¯ :: Pokemon hack no root generate PokeBal.ls | iOS-Android', 1771, 1, 1468432988, 1468432988, 0, 0, 0, 0, 0, 0),
(27017, 27027, 'author', 'en', '♍{*❶❶❶*} :: Pokemon hack cheats generate PokeCoins, PokeBalls', 1771, 1, 1468432991, 1468432991, 0, 0, 0, 0, 0, 0),
(27018, 27028, 'author', 'en', '♐2016♐ 13/07/2016 :: Pokemon tricheurs ', 1771, 1, 1468432995, 1468432995, 0, 0, 0, 0, 0, 0),
(27019, 27029, 'author', 'en', '♎[![GETHACK]!]♎ :: Pokemon Hack Deutsch PokeCoin,s | 2016', 1771, 1, 1468433002, 1468433002, 0, 0, 0, 0, 0, 0),
(27020, 27030, 'author', 'en', '♓#❹❹❹♓ :: ch♊eat Pokemon ', 1771, 1, 1468433004, 1468433004, 0, 0, 0, 0, 0, 0),
(27021, 27031, 'author', 'en', '✓|_/¯HA*CKPACK¯\\_| 13..07.2016 :: Hack Para Pokemon generate PokeBalls | 2016', 1771, 1, 1468433012, 1468433012, 0, 0, 0, 0, 0, 0),
(27022, 27032, 'author', 'en', '►(!❼❼❼!)▻ :: Pokemon hack for android generate PokeBalls | Android/iOS', 1771, 1, 1468433014, 1468433014, 0, 0, 0, 0, 0, 0),
(27023, 27033, 'author', 'en', '☑{*hacK*}☒ 13/07/2016 :: Pokemon cheat code entry ', 1771, 1, 1468433026, 1468433026, 0, 0, 0, 0, 0, 0),
(27024, 27034, 'author', 'en', '♇{{❶❶❶}} :: Pokemon hack To.ol download PokeCoins', 1771, 1, 1468433028, 1468433028, 0, 0, 0, 0, 0, 0),
(27025, 27035, 'author', 'en', '♿[!❽❽�;�!]♿ :: Pokemon Hack Gratis h.ack PokeCoins | Androi.d|/iOS', 1771, 1, 1468433035, 1468433035, 0, 0, 0, 0, 0, 0),
(27026, 27036, 'author', 'en', '♴[!Hac;kgenerator!]♴ :: Pokemon cheats real hack PokeCoins, PokeBalls', 1771, 1, 1468433040, 1468433040, 0, 0, 0, 0, 0, 0),
(27027, 27037, 'author', 'en', '☧(!FULLHACK!)☨ 13:07:2016 :: Pokemon tricheurs ', 1771, 1, 1468433063, 1468433063, 0, 0, 0, 0, 0, 0),
(27028, 27038, 'author', 'en', '✿❶❶❶✿ :: Pokemon cheat that works ', 1771, 1, 1468433065, 1468433065, 0, 0, 0, 0, 0, 0),
(27029, 27039, 'author', 'en', '☜(~FULLHACK~)☜ 13.07.2016 :: Pokemon cheats that actually work generate Pok▦eBalls | 2016', 1771, 1, 1468433075, 1468433075, 0, 0, 0, 0, 0, 0),
(27030, 27040, 'author', 'en', '☷❸❸❸☸ :: Pokemon cheats real ', 1771, 1, 1468433081, 1468433081, 0, 0, 0, 0, 0, 0),
(27031, 27041, 'author', 'en', '◔❻❻❻◔ ::, Pokemon hack no download  ◼', 1771, 1, 1468433084, 1468433084, 0, 0, 0, 0, 0, 0),
(27032, 27042, 'author', 'en', '▧[~[GETHACK]~]▧ 13.07.2016 :: Pokemon ipa h.ack ', 1771, 1, 1468433090, 1468433090, 0, 0, 0, 0, 0, 0),
(27033, 27043, 'author', 'en', '♐[![❽❽❽]!]♐ :: Pokemon Online Generator ', 1771, 1, 1468433090, 1468433090, 0, 0, 0, 0, 0, 0),
(27034, 27044, 'author', 'en', '❝#CHEAT❝ :: Pokemon cheats and^ t.ips hack PokeCoins | iOS-Android', 1771, 1, 1468433095, 1468433095, 0, 0, 0, 0, 0, 0),
(27035, 27045, 'author', 'en', '☭❷❷❷☭ :: c^heat Pokemon free PokeBalls', 1771, 1, 1468433106, 1468433106, 0, 0, 0, 0, 0, 0),
(27036, 27046, 'author', 'en', '❞~!~❺;❺❺❟ :: Pokemon cheat ios ', 1771, 1, 1468433106, 1468433106, 0, 0, 0, 0, 0, 0),
(27037, 27047, 'author', 'en', '▭(!(13.07.◼2016)!) :: Hack Para Pokemon hack PokECoins, PokeBalls | Andr◸oid/iOS/', 1771, 1, 1468433111, 1468433111, 0, 0, 0, 0, 0, 0),
(27038, 27048, 'author', 'en', '☇(*(�:���☧���.�)*) :: cheats for Pokemon h♛ack PokeBalls | iOS-Andr✰oi,d', 1771, 1, 1468433112, 1468433112, 0, 0, 0, 0, 0, 0),
(27039, 27049, 'author', 'en', '◄_/¯❼❼❼¯\\_ :: Pokemon Hack No Survey PokeCoins | AndRoid', 1771, 1, 1468433117, 1468433117, 0, 0, 0, 0, 0, 0),
(27040, 27050, 'author', 'en', '◎(~(fullhack)~)◎ 13.07.20*16 :: Pokemon Juwelen Hack ', 1771, 1, 1468433128, 1468433128, 0, 0, 0, 0, 0, 0),
(27041, 27051, 'author', 'en', '✗[*mod*]✗ 13-07-2016 :: Pokemon Cheats free PokeCoins | Android/iOS', 1771, 1, 1468433130, 1468433130, 0, 0, 0, 0, 0, 0),
(27042, 27052, 'author', 'en', '☷{{HAC✈KCHEATS}}☸ 13:!07:201♝6 :: Pokemon Hack Cheat Juwelen Deutsch! free PokeCoins, PokeB❜alls | iOS-An▰droid', 1771, 1, 1468433135, 1468433135, 0, 0, 0, 0, 0, 0),
(27043, 27053, 'author', 'en', '☊#HACKED 13.07.2016 :: Pokemon Hack iphone hack PokeBalls | iOS-Android', 1771, 1, 1468433142, 1468433142, 0, 0, 0, 0, 0, 0),
(27044, 27054, 'author', 'en', '♛(!(�.��❸❸)!)♜ :: how to get hack tool Pokemon ', 1771, 1, 1468433152, 1468433152, 0, 0, 0, 0, 0, 0),
(27045, 27055, 'author', 'en', '☀[*[mod]*]☀ 13-07-2016 :: Pokemon cheats too☢l ', 1771, 1, 1468433157, 1468433157, 0, 0, 0, 0, 0, 0),
(27046, 27056, 'author', 'en', '♅|_/¯CHEATS¯\\_|♅ 13:07:2016 :.:. Pokemon hack software ', 1771, 1, 1468433157, 1468433157, 0, 0, 0, 0, 0, 0),
(27047, 27057, 'author', 'en', '☼❾❾❾☼ :: Pokemon hack generator generate PokeBalls', 1771, 1, 1468433166, 1468433166, 0, 0, 0, 0, 0, 0),
(27048, 27058, 'author', 'en', '✉(~cheats~)✉ 13:07:2016 :: Pokemon Glitch and Glitches ', 1771, 1, 1468433176, 1468433176, 0, 0, 0, 0, 0, 0),
(27049, 27059, 'author', 'en', '☟❻❻❻♡ :: Pokemon c▱heats online ', 1771, 1, 1468433182, 1468433182, 0, 0, 0, 0, 0, 0),
(27050, 27060, 'author', 'en', '☀{~{❸�.�❸}~} :: how to h\\ac:k Pokemon free PokeBalls | Android/iOS', 1771, 1, 1468433185, 1468433185, 0, 0, 0, 0, 0, 0),
(27051, 27061, 'author', 'en', '◰20/16◰ :: Pokemon Online Generator PokeCoins | Android', 1771, 1, 1468433188, 1468433188, 0, 0, 0, 0, 0, 0),
(27052, 27062, 'author', 'en', '❄[[�;��❻❻]]❄ :: Pokemon hack n.o jaibreak ', 1771, 1, 1468433216, 1468433216, 0, 0, 0, 0, 0, 0),
(27053, 27063, 'author', 'en', '❅(!(h.a.c.k.g.e.n.e.r.a.t.o.r.)!) 13/.07.2016 :: Pokemon Juwelen Hack hack PokeCoins | iO◻S-Android', 1771, 1, 1468433238, 1468433238, 0, 0, 0, 0, 0, 0),
(27054, 27064, 'author', 'en', '☒$hAc♂kcheats☓ 13-07-2016 :: Pokemon Hack Cheat Ju✗weleN Deutsch! free PokeCoins, PokeBalls | iOS-Android☴', 1771, 1, 1468433241, 1468433241, 0, 0, 0, 0, 0, 0),
(27055, 27065, 'author', 'en', '®�♢�❺❺® :: AStucEs Pour Pokemon hack PokeCoins | An.droid', 1771, 1, 1468433242, 1468433242, 0, 0, 0, 0, 0, 0),
(27056, 27066, 'author', 'en', '☳[~[fullhack]~] 13-07-2016 :: Pokemon Hack Ch.ea▸ts Trainer PokeBallS | Android/iOS', 1771, 1, 1468433242, 1468433242, 0, 0, 0, 0, 0, 0),
(27057, 27067, 'author', 'en', '☔{!{❽❽❽}!}☕ :: Pokemon cheats reaL Hack PokeCoins | 2016', 1771, 1, 1468433246, 1468433246, 0, 0, 0, 0, 0, 0),
(27058, 27068, 'author', 'en', '☷{HACKCHEATS} 13.07.2016 :: Pokemon c◶heats and h☦acks hack Pok❱eCoin:s | Android', 1771, 1, 1468433260, 1468433260, 0, 0, 0, 0, 0, 0),
(27059, 27069, 'author', 'en', '◀(~(�☁�❾❾)~) :: Pokemon cheats codes ', 1771, 1, 1468433264, 1468433264, 0, 0, 0, 0, 0, 0),
(27060, 27070, 'author', 'en', '☳[![❶❶❶]!]☴ ::\\ Pokemon pirater telecharger PokeBall|s | 2016', 1771, 1, 1468433265, 1468433265, 0, 0, 0, 0, 0, 0),
(27061, 27071, 'author', 'en', '♙{*{❶❶❶}*}♚ :: Pokemon ipa haCk free PokeBalls', 1771, 1, 1468433266, 1468433266, 0, 0, 0, 0, 0, 0),
(27062, 27072, 'author', 'en', '☿{!hacked!}☿ 13/07/2016 :: Pokemon hack tool online ', 1771, 1, 1468433270, 1468433270, 0, 0, 0, 0, 0, 0),
(27063, 27073, 'author', 'en', '◲[![❽�.�❽]!]◳ :: Pokemon Hack 2016 free PokeBalls | Android', 1771, 1, 1468433282, 1468433282, 0, 0, 0, 0, 0, 0),
(27064, 27074, 'author', 'en', '♽¯\\_❻❻❻_/¯♾ :: Pokemon c^h^e^a^t^ Codes PokeCoins | iOS-Android', 1771, 1, 1468433287, 1468433287, 0, 0, 0, 0, 0, 0),
(27065, 27075, 'author', 'en', '✨|_/¯❸❸❸¯\\_|✨ :: Pokemon c!h!e!a!t! revieW gen♃erate PokeCoins, P.o.k.e.B.a.l.l.s. | Android/iOS', 1771, 1, 1468433289, 1468433289, 0, 0, 0, 0, 0, 0),
(27066, 27076, 'author', 'en', '❧{~{H.A.C.K.E.D.}~}★ 13:..07:2,016 :: Pokemon hack proof free P.o.k.e.B.a.l.l.s. | iOS-Android', 1771, 1, 1468433290, 1468433290, 0, 0, 0, 0, 0, 0),
(27067, 27077, 'author', 'en', '❘{*{❹❹❹}*} :: Pokemon tips for noobs free PokeCoins', 1771, 1, 1468433294, 1468433294, 0, 0, 0, 0, 0, 0),
(27068, 27078, 'author', 'en', '✯{!{❹�.�❹}!} :: Pokemon hack android* free PokeCoins | Android○/\\iOS', 1771, 1, 1468433302, 1468433302, 0, 0, 0, 0, 0, 0),
(27069, 27079, 'author', 'en', '◘{!{❽❽❽}!}◘ :: Pokemon Online Hack ', 1771, 1, 1468433308, 1468433308, 0, 0, 0, 0, 0, 0),
(27070, 27080, 'author', 'en', '■[!❸�..��❸!] :: Pokemon cheats for ipad free PokeBalls | iOS-Android', 1771, 1, 1468433311, 1468433311, 0, 0, 0, 0, 0, 0),
(27071, 27081, 'author', 'en', '◃|_/¯❶❶❶¯\\_|◃ :: Astuces Pour Pokemon ', 1771, 1, 1468433313, 1468433313, 0, 0, 0, 0, 0, 0),
(27072, 27082, 'author', 'en', '◞[!❾❾�,�!]◟ :: Pokemon cheats tool hack Po◔keCoins | iOS-Androi.d', 1771, 1, 1468433337, 1468433337, 0, 0, 0, 0, 0, 0),
(27073, 27083, 'author', 'en', '☄{*{❺❺❺}*}☇ :: Pokemon c\\h\\e\\a\\t\\ c,odes generate Pok:eBalls | Android', 1771, 1, 1468433338, 1468433338, 0, 0, 0, 0, 0, 0),
(27074, 27084, 'author', 'en', '◱{*{ME.GAHACK}*} 1/3.07.2016 :: Pokemon hack mod apk generate PokeCoins,❘ PokeBalls | 2016', 1771, 1, 1468433340, 1468433340, 0, 0, 0, 0, 0, 0),
(27075, 27085, 'author', 'en', '◘~!~❶�♡��❶◙ :: Telecharger Pokemon Hack Gratuit ', 1771, 1, 1468433341, 1468433341, 0, 0, 0, 0, 0, 0),
(27076, 27086, 'author', 'en', '◢[*[CHEAT]*] 13-07-2016 :: Pokemon on google play doWnload  Hack PokeBal☍ls | 2016', 1771, 1, 1468433365, 1468433365, 0, 0, 0, 0, 0, 0),
(27077, 27087, 'author', 'en', '♛CHEATS,♛ 13-07-201.◑6 :: Pokemon Hacke.n .', 1771, 1, 1468433385, 1468433385, 0, 0, 0, 0, 0, 0),
(27078, 27088, 'author', 'en', '♓[!m;o;d;!] 13:07:2016 :: Pokemon cheaTs◳ that actually work free PokECoiNs | 2016', 1771, 1, 1468433389, 1468433389, 0, 0, 0, 0, 0, 0),
(27079, 27089, 'author', 'en', '◰[*[❹❹,❹]*]◰ ::  generate P^okeCoins |. 2016', 1771, 1, 1468433392, 1468433392, 0, 0, 0, 0, 0, 0),
(27080, 27090, 'author', 'en', '✦(*hacken◣gine*) :: Pokemon Hack C|ydia free PokeBalls | iOS-Android', 1771, 1, 1468433399, 1468433399, 0, 0, 0, 0, 0, 0),
(27081, 27091, 'author', 'en', '☰((ENGINE))☰ 13/07/2016 :: Pokemon cheat download Generate PokeBalls | Android', 1771, 1, 1468433402, 1468433402, 0, 0, 0, 0, 0, 0),
(27082, 27092, 'author', 'en', '○mod○ 13-07-2016 :: Pokemon hack ifile generate P..o..k..e..B..a..l..l..s..', 1771, 1, 1468433420, 1468433420, 0, 0, 0, 0, 0, 0),
(27083, 27093, 'author', 'en', '◆{*{❺❺❺}*}◇ :: Pokemon Hack Cydia ', 1771, 1, 1468433422, 1468433422, 0, 0, 0, 0, 0, 0),
(27084, 27094, 'author', 'en', '❀{!{HACKPAC▽K}!}❃ 13.07.2016 :: Pokemon hack no survey  free PokeBalls | iOS-Android', 1771, 1, 1468433424, 1468433424, 0, 0, 0, 0, 0, 0),
(27085, 27095, 'author', 'en', '✫(*geNerator*) 13-07-2016 :: Pokemon Hack Cheats Trainer generate PokeBalls | iOS-Android', 1771, 1, 1468433424, 1468433424, 0, 0, 0, 0, 0, 0),
(27086, 27096, 'author', 'en', '☙gethack 13.07.2016 :: Pokemon hack tool free PokeCoins, PokeBalls', 1771, 1, 1468433424, 1468433424, 0, 0, 0, 0, 0, 0),
(27087, 27097, 'author', 'en', '▲((�❱��.�❼))△ :: free game hac☣k tools Pokemon PokeCoins, PokeBalls | Android', 1771, 1, 1468433442, 1468433442, 0, 0, 0, 0, 0, 0),
(27088, 27098, 'author', 'en', '☁{!{❾❾❾}!}☁ :: Pokemon hack that wo◼rks generate PokeBalls | iOS-Android', 1771, 1, 1468433444, 1468433444, 0, 0, 0, 0, 0, 0),
(27089, 27099, 'author', 'en', '☚HACK :: Pokemon cheat tool hack PokeCoins | Android', 1771, 1, 1468433447, 1468433447, 0, 0, 0, 0, 0, 0),
(27090, 27100, 'author', 'en', '☗{❸❸❸}☗ :: Pokemon Cheats 2^0^1^6^ free PokeCoins | Android', 1771, 1, 1468433448, 1468433448, 0, 0, 0, 0, 0, 0),
(27091, 27101, 'author', 'en', '◤#HACKED 13.07.2*016 :: Pokemon hack no jaibreak fre.e PokeCoins | iO..S-Android', 1771, 1, 1468433452, 1468433452, 0, 0, 0, 0, 0, 0),
(27092, 27102, 'author', 'en', '☺(!❶❶❶!) :: Pokemon Che,ats ', 1771, 1, 1468433464, 1468433464, 0, 0, 0, 0, 0, 0),
(27093, 27103, 'author', 'en', '○G..E..T..H..A..C..K..◌ 13-07-2016 :: Pokemon cheat sheet ', 1771, 1, 1468433465, 1468433465, 0, 0, 0, 0, 0, 0),
(27094, 27104, 'author', 'en', '♸_/¯❸❸�;�¯\\_♸ :: cheat Pokemon PokeBalls', 1771, 1, 1468433469, 1468433469, 0, 0, 0, 0, 0, 0),
(27095, 27105, 'author', 'en', '❁07.13..2016❁ :: Pokemon Glitch ', 1771, 1, 1468433471, 1468433471, 0, 0, 0, 0, 0, 0),
(27096, 27106, 'author', 'en', '♍#❶❶❶♎ :: Pokemon ha,ck down\\lo.Ad PokeCoins | 2016', 1771, 1, 1468433475, 1468433475, 0, 0, 0, 0, 0, 0),
(27097, 27107, 'author', 'en', '♣[ha★cktool]♣ 13/07/2016 :: Pokemon GlitCh generate PokeCo.ins, PokeBalls | AnDroid', 1771, 1, 1468433485, 1468433485, 0, 0, 0, 0, 0, 0),
(27098, 27108, 'author', 'en', '♨###�◗�◉�❷❷♩ :: Pokemon cheat code entrY generate PokeCoins, PokeBalls | 2016', 1771, 1, 1468433487, 1468433487, 0, 0, 0, 0, 0, 0),
(27099, 27109, 'author', 'en', '◥[~❺❺❺~]◦ :: Pokemon hack ipad hac☢k PokeBalls | 2016', 1771, 1, 1468433493, 1468433493, 0, 0, 0, 0, 0, 0),
(27100, 27110, 'author', 'en', '♭###❷❷❷♭ :: Pokemon tips! advanced P.okeCoins | An|droI♕d/iOS', 1771, 1, 1468433498, 1468433498, 0, 0, 0, 0, 0, 0),
(27101, 27111, 'author', 'en', '☹[*[❼❼^❼]*]☹ :: Pokemon tips advanced free PokeBalls | Android', 1771, 1, 1468433524, 1468433524, 0, 0, 0, 0, 0, 0),
(27102, 27112, 'author', 'en', '✉HACKED 13:07:2016 :: Pokemon cheats android PokeBaLls', 1771, 1, 1468433534, 1468433534, 0, 0, 0, 0, 0, 0),
(27103, 27113, 'author', 'en', '◒fullhack :: Pokemon hack jaibreak free Poke❊Coins, PokeBalls | iOS-Android;', 1771, 1, 1468433534, 1468433534, 0, 0, 0, 0, 0, 0),
(27104, 27114, 'author', 'en', '◽(!mod!)◾ 13:07:2016 :: Pokemon cheat tool hack PokeCoins, PokeBalls', 1771, 1, 1468433542, 1468433542, 0, 0, 0, 0, 0, 0),
(27105, 27115, 'author', 'en', '❀(*(❻❻❻)*) :: Pokemon tips for beginn▯ers ge/nerate PokeBalls | Androi♑d', 1771, 1, 1468433548, 1468433548, 0, 0, 0, 0, 0, 0),
(27106, 27116, 'author', 'en', '▶❻❻❻▷ :: Pokemon hack work free PokeBalls | iOS-Android', 1771, 1, 1468433549, 1468433549, 0, 0, 0, 0, 0, 0),
(27107, 27117, 'author', 'en', '◻~!~megaHack◼ :: Pokemon Ha☔ck Cheats Trainer ', 1771, 1, 1468433556, 1468433556, 0, 0, 0, 0, 0, 0),
(27108, 27118, 'author', 'en', '♖$❶❶�.��♗ :: Pokemon hack no jaibreak free PokeCoins, PokeBalls | iOS-Android', 1771, 1, 1468433557, 1468433557, 0, 0, 0, 0, 0, 0),
(27109, 27119, 'author', 'en', '♥�.��❼❼♦ :: Pokemon hac|k codes free PokeBalls | Android', 1771, 1, 1468433564, 1468433564, 0, 0, 0, 0, 0, 0),
(27110, 27120, 'author', 'en', '[[.*.EsPn^TV**..]] Crusaders F.C. vs København L.i.v.e S.t.r.e.a.m', 2057, 1, 1468433584, 1468433584, 0, 0, 0, 0, 0, 0),
(27111, 27121, 'author', 'en', '[[.*.EsPn^TV**..]] Olimpija NK vs Trenčín L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468434111, 1468434111, 0, 0, 0, 0, 0, 0),
(27112, 27122, 'author', 'en', 'Columbus Crew vs Toronto FC', 2075, 1, 1468434290, 1468434290, 0, 0, 0, 0, 0, 0),
(27113, 27123, 'author', 'en', '>>Live%%~Columbus Crew vs Toronto FC Live MLS Soccer Pc tv', 2075, 1, 1468434552, 1468434783, 0, 0, 0, 0, 0, 0),
(27114, 27124, 'author', 'en', '[[.*.EsPn^TV**..]] Chester FC vs Wigan Athletic  L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468434942, 1468434942, 0, 0, 0, 0, 0, 0),
(27115, 27125, 'author', 'en', '[[Soccer~Live]]Chester vs Wigan Athletic Live Stream', 2074, 1, 1468435099, 1468435253, 0, 0, 0, 0, 0, 0),
(27116, 27126, 'author', 'en', '[[.*.EsPn^TV**..]] Cardiff City vs Forest Green Rovers  L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468435471, 1468435471, 0, 0, 0, 0, 0, 0),
(27117, 27127, 'author', 'en', 'jems', 2077, 1, 1468435697, 1468435697, 0, 0, 0, 0, 0, 0),
(27118, 27128, 'author', 'en', 'broadcasT|$|Mcdonald vs Lineker L.i.v.e  O.n.l.i.n.e Full Coverage Ultimate Fight 91 Tv Streaming', 2079, 1, 1468435733, 1468436176, 0, 0, 0, 0, 0, 0),
(27119, 27129, 'author', 'en', 'Cok+>>>Columbus Crew vs Toronto FC Live Strea-m Online', 2077, 1, 1468435832, 1468435832, 0, 0, 0, 0, 0, 0),
(27120, 27130, 'author', 'en', '[[.*.EsPn^TV**..]]Dundalk vs FH Hafnarfjordur L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468435863, 1468435863, 0, 0, 0, 0, 0, 0),
(27121, 27131, 'author', 'en', 'En-Viv0}}}} Mcdonald vs Lineker L.i.v.e O.n.l.i.n.e Full Coverage Ultimate Fight 91 Tv Streaming', 2079, 1, 1468436283, 1468437299, 0, 0, 0, 0, 0, 0),
(27122, 27132, 'author', 'en', 'sdfsffg', 2081, 1, 1468436591, 1468436591, 0, 0, 0, 0, 0, 0),
(27123, 27133, 'author', 'en', '[[.*.EsPn^TV**..]]Ipswich Town vs Barnet L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468436659, 1468436659, 0, 0, 0, 0, 0, 0),
(27124, 27134, 'author', 'en', 'Run+>>New York Cosmos vs Jacksonville Armada live Soccer', 2077, 1, 1468436772, 1468436772, 0, 0, 0, 0, 0, 0),
(27125, 27135, 'author', 'en', '[[.*.EsPn^TV**..]]Mystics vs Mercury Basketball  L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468437074, 1468437074, 0, 0, 0, 0, 0, 0),
(27126, 27136, 'author', 'en', '(LIVE)McDonald vs Lineker Live UFC FIGHT TODAY', 2077, 1, 1468437194, 1468437194, 0, 0, 0, 0, 0, 0),
(27127, 27137, 'author', 'en', '!!>LiVe!!> Sela vs Ram Live Strea.m Online Gam.e P.c', 2082, 1, 1468438791, 1468438894, 0, 0, 0, 0, 0, 0),
(27128, 27138, 'author', 'en', '!!@BBc^Tv!!> Sela vs Ram Live Stream Gam.e {Tennis} PC T.v', 2082, 1, 1468439002, 1468439361, 0, 0, 0, 0, 0, 0),
(27129, 27139, 'author', 'en', '+>^COnTEANE::UFC*91:McDonald vs Lineker live stream 14.07.2016', 2083, 1, 1468439227, 1468439227, 0, 0, 0, 0, 0, 0),
(27130, 27140, 'author', 'en', 'Movies.HD) WATCH The Secret Life of Pets Online Free HD Streaming', 2070, 1, 1468439231, 1468439231, 0, 0, 0, 0, 0, 0),
(27131, 27141, 'author', 'en', '[Watch GAA]  The Secret Life of Pets Live Stream Online', 2070, 1, 1468439385, 1468439385, 0, 0, 0, 0, 0, 0),
(27132, 27142, 'author', 'en', 'En-VOvI}}}} Mcdonald vs Lineker L.i.v.e O.n.l.i.n.e Full C.overage U.ltimate F.ight 91 T.v S.tream 14.07.2016', 2083, 1, 1468439462, 1468439462, 0, 0, 0, 0, 0, 0),
(27133, 27143, 'author', 'en', '[Watch GAA] Galway vs Kilkenny Live Stream Onlinee', 2070, 1, 1468439500, 1468439500, 0, 0, 0, 0, 0, 0),
(27134, 27144, 'author', 'en', 'WATCH GAA@ Star Trek Vs Beyond live Leinster Hurling-Final TV', 2070, 1, 1468439668, 1468439668, 0, 0, 0, 0, 0, 0),
(27135, 27145, 'author', 'en', 'UFC||liVe||M.cDonald v/s Lineker live stream 2016.07.14', 2083, 1, 1468439684, 1468439684, 0, 0, 0, 0, 0, 0),
(27136, 27146, 'author', 'en', 'WATCH GAAAA@ Galway Vs Kilkenny live Leinster Hurling-Final TV', 2070, 1, 1468439795, 1468439795, 0, 0, 0, 0, 0, 0),
(27137, 27147, 'author', 'en', 'Watch The Secret Life of Pets Online Free Putlocker ', 2070, 1, 1468440004, 1468440431, 0, 0, 0, 0, 0, 0),
(27138, 27148, 'author', 'en', 'LIVE TV::New York Red Bulls vs Orlando City live Strea-m Online Coverge...', 2077, 1, 1468440240, 1468440291, 0, 0, 0, 0, 0, 0),
(27139, 27149, 'author', 'en', 'Watch The Purge: Election Year Online Free Putlocker', 2070, 1, 1468440510, 1468440807, 0, 0, 0, 0, 0, 0),
(27140, 27150, 'author', 'en', 'McDonald vs Lineker Live', 2081, 1, 1468440576, 1468440576, 0, 0, 0, 0, 0, 0),
(27141, 27151, 'author', 'en', 'Watch The Purge: Election Year Full Movie Online (2016) Free', 2070, 1, 1468440853, 1468441160, 0, 0, 0, 0, 0, 0),
(27142, 27152, 'author', 'en', 'Watch Ice Age: Collision Course Online Free Putlocker', 2070, 1, 1468441303, 1468441632, 0, 0, 0, 0, 0, 0),
(27143, 27153, 'author', 'en', '[[.*.EsPn^TV**..]]2016 ESPY Awards red carpet L.i.v.e S.t.r.e.a.m ', 2057, 1, 1468441381, 1468441381, 0, 0, 0, 0, 0, 0),
(27144, 27154, 'author', 'en', 'UFC Fight Night 91 live stream', 2081, 1, 1468441788, 1468441788, 0, 0, 0, 0, 0, 0),
(27145, 27155, 'author', 'en', 'HD~TV~New York Red Bulls vs Orlando City SC L.i.v.e St-re-am', 2072, 1, 1468442526, 1468442526, 0, 0, 0, 0, 0, 0),
(27146, 27156, 'author', 'en', 'UFC 91 Devin Clark vs Alex Nicholson Live stream', 2081, 1, 1468442643, 1468442643, 0, 0, 0, 0, 0, 0),
(27147, 27157, 'author', 'en', 'Watch The Angry Birds Movie Online Free Full Putlockers', 2085, 1, 1468442831, 1468442831, 0, 0, 0, 0, 0, 0),
(27148, 27158, 'author', 'en', 'UFC 91 Rani Yahya vs Matthew Lopez Live stream', 2081, 1, 1468442910, 1468442910, 0, 0, 0, 0, 0, 0),
(27149, 27159, 'author', 'en', 'HD~TV~Columbus Crew SC vs Toronto FC L.i.v.e St-re-am', 2072, 1, 1468443417, 1468443417, 0, 0, 0, 0, 0, 0),
(27150, 27160, 'author', 'en', 'UFC 91Scott Holtzman vs Cody Pfister Live stream', 2081, 1, 1468443418, 1468443418, 0, 0, 0, 0, 0, 0),
(27151, 27161, 'author', 'en', 'UFC||liVe||M.cDonald v/s Lineker live stream 2016.07.14 | Medici', 2086, 1, 1468443449, 1468443496, 0, 0, 0, 0, 0, 0),
(27152, 27162, 'author', 'en', 'UFC 91 Scott Holtzman vs Cody Pfister Live stream', 2081, 1, 1468443700, 1468443700, 0, 0, 0, 0, 0, 0),
(27153, 27163, 'author', 'en', 'HD~TV~UFC Fight Night MAcDonald vs Lineker L.i.v.e St-re-am', 2072, 1, 1468443840, 1468443840, 0, 0, 0, 0, 0, 0),
(27154, 27164, 'author', 'en', 'UFC||liVe||M.cDonald v/s Lineker live stream 2016.07.14 | Medicin', 2086, 1, 1468445662, 1468445662, 0, 0, 0, 0, 0, 0),
(27155, 27165, 'author', 'en', '+>^COnTEANE::UFC*91:McDonald vs Lineker live stream 14.07', 2086, 1, 1468447287, 1468447287, 0, 0, 0, 0, 0, 0),
(27156, 27166, 'author', 'en', 'UFC 91 Eric Spicely vs Sam Alvey Live stream', 2081, 1, 1468447777, 1468447777, 0, 0, 0, 0, 0, 0),
(27157, 27167, 'author', 'en', 'UFC 91 Lauren Murphy vs Katlyn Chookagian Live stream', 2081, 1, 1468448016, 1468448016, 0, 0, 0, 0, 0, 0),
(27158, 27168, 'author', 'en', 'UFC Fight Night 91 Michael McDonald vs John Lineker Live stream', 2081, 1, 1468448282, 1468448282, 0, 0, 0, 0, 0, 0),
(27159, 27169, 'author', 'en', 'Run+>>>McDonald vs Lineker Live S,t,r,e,a,m onli,n,e 12/07/201', 2086, 1, 1468448540, 1468448540, 0, 0, 0, 0, 0, 0),
(27160, 27170, 'author', 'en', 'Michael McDonald vs John Lineker Live stream', 2081, 1, 1468448923, 1468448923, 0, 0, 0, 0, 0, 0),
(27161, 27171, 'author', 'en', 'LIVE HDQ::Columbus Crew vs Toronto FC Live Strea-m Online', 2077, 1, 1468450241, 1468450241, 0, 0, 0, 0, 0, 0),
(27162, 27172, 'author', 'en', '[*TV*+] ESPYs Red Carpet Live ..St.ream -... 13 July 2016', 2057, 1, 1468450664, 1468450823, 0, 0, 0, 0, 0, 0),
(27163, 27173, 'author', 'en', 'Tony Ferguson vs Landon Vannata Live stream', 2081, 1, 1468451026, 1468451026, 0, 0, 0, 0, 0, 0),
(27164, 27174, 'author', 'en', '+>^COnTEANE::UFC*91:McDonald vs Lineker live stream 14.', 2086, 1, 1468451455, 1468451455, 0, 0, 0, 0, 0, 0),
(27165, 27175, 'author', 'en', 'COnTEANE::UFC*91:McDonald vs Lineker live stream', 2098, 1, 1468452026, 1468452026, 0, 0, 0, 0, 0, 0),
(27166, 27176, 'author', 'en', '[Live-Ufc]McDonald vs Lineker Live Online UFC Fight Night 91 S-', 2086, 1, 1468452162, 1468452162, 0, 0, 0, 0, 0, 0),
(27167, 27177, 'author', 'en', 'f.I.g.H.t>> McDonald vs Lineker L.i.v.e s.t.r.e.a.m.i.n.g 7/13/2016', 2098, 1, 1468452210, 1468452210, 0, 0, 0, 0, 0, 0),
(27168, 27178, 'author', 'en', 'Watch Suits Season 6 Episode 1 Online', 2098, 1, 1468452436, 1468452436, 0, 0, 0, 0, 0, 0),
(27169, 27179, 'author', 'en', 'Suits Season 6 Episode 1 Online Free', 2098, 1, 1468452592, 1468452592, 0, 0, 0, 0, 0, 0),
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(34171, 34181, 'author', 'en', 'Viitorul 2 Constanța vs Unirea Tărlungeni Live Stream', 2235, 1, 1473818582, 1473818582, 0, 0, 0, 0, 0, 0),
(34172, 34182, 'author', 'en', 'Watch Bayer Leverkusen U19 v CSKA Moscow U19 UEFA Youth League Live', 2125, 1, 1473818662, 1473818662, 0, 0, 0, 0, 0, 0),
(34175, 34185, 'author', 'en', 'Watch Al-Ahed vs Al-Muharraq AFC Cup Live September 14, 2016', 2232, 1, 1473822219, 1473822219, 0, 0, 0, 0, 0, 0),
(34176, 34186, 'author', 'en', 'Watch Salavat Yulaev vs Admiral hockey Live 14.09.2016', 2125, 1, 1473822792, 1473822792, 0, 0, 0, 0, 0, 0),
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(34193, 34203, 'author', 'en', 'Fondi v Akragas Italian Lega Pro Group C Live Stream', 2125, 1, 1473830616, 1473830616, 0, 0, 0, 0, 0, 0),
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(34200, 34210, 'author', 'en', 'Viitorul 2 v Tărlungeni Romanian Cup Live Stream September 14, 2016', 2232, 1, 1473833319, 1473833319, 0, 0, 0, 0, 0, 0),
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(34232, 34242, 'author', 'en', 'Watch Pineros de Isla de la Juventud - Tigres de Ciego de Ávila live streaming 14.09.2016', 2240, 1, 1473838327, 1473838327, 0, 0, 0, 0, 0, 0),
(34236, 34246, 'author', 'en', 'Watch Al-Nasr vs Al-Jaish AFC Champions League live streaming September 14, 2016', 2239, 1, 1473838647, 1473838647, 0, 0, 0, 0, 0, 0),
(34237, 34247, 'author', 'en', 'Watch Al-Nasr v Al-Jaish AFC Champions League Live September 14, 2016', 2235, 1, 1473838842, 1473838842, 0, 0, 0, 0, 0, 0),
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(34245, 34255, 'author', 'en', 'Live streaming Neftekhimik v Lokomotiv hockey tv watch September 14, 2016', 2232, 1, 1473839235, 1473839235, 0, 0, 0, 0, 0, 0),
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(34251, 34261, 'author', 'en', 'Watch Central Ballester vs Lugano soccer Live', 2232, 1, 1473840047, 1473840047, 0, 0, 0, 0, 0, 0),
(34252, 34262, 'author', 'en', 'Live streaming Lauren Davis vs Samantha Crawford tv watch 14.09.2016', 2240, 1, 1473840071, 1473840071, 0, 0, 0, 0, 0, 0),
(34254, 34264, 'author', 'en', 'Live streaming Flensburg-Handewitt vs Magdeburg German Bundesliga Handball tv watch September 14, 2016', 2235, 1, 1473840210, 1473840210, 0, 0, 0, 0, 0, 0),
(34256, 34266, 'author', 'en', 'Watch Brugge - Leicester soccer Live 9/14/2016', 2240, 1, 1473840269, 1473840269, 0, 0, 0, 0, 0, 0),
(34258, 34268, 'author', 'en', 'Live streaming Bayer Leverkusen - CSKA Moscow tv watch September 14, 2016', 2125, 1, 1473840518, 1473840518, 0, 0, 0, 0, 0, 0),
(34262, 34272, 'author', 'en', 'Bækkelagets vs Bodø Live Stream 14.09.2016', 2235, 1, 1473840576, 1473840576, 0, 0, 0, 0, 0, 0),
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(34268, 34278, 'author', 'en', 'Al-Nasr - Al-Jaish Live Stream September 14, 2016', 2232, 1, 1473841176, 1473841176, 0, 0, 0, 0, 0, 0),
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(34272, 34282, 'author', 'en', 'Legia v Borussia D soccer Live Stream 14.09.2016', 2125, 1, 1473841547, 1473841547, 0, 0, 0, 0, 0, 0),
(34274, 34284, 'author', 'en', 'Live streaming Vibonese vs Matera Italian Lega Pro Group C tv watch September 14, 2016', 2232, 1, 1473841702, 1473841702, 0, 0, 0, 0, 0, 0),
(34275, 34285, 'author', 'en', 'Live streaming Fondi v Akragas tv watch 14.09.2016', 2239, 1, 1473841807, 1473841807, 0, 0, 0, 0, 0, 0),
(34278, 34288, 'author', 'en', 'Watch Chicago Cubs - St. Louis Cardinals livestream', 2239, 1, 1473842094, 1473842094, 0, 0, 0, 0, 0, 0),
(34282, 34292, 'author', 'en', 'Live streaming Tampa Bay v Toronto  September 14, 2016', 2240, 1, 1473842282, 1473842282, 0, 0, 0, 0, 0, 0),
(34283, 34293, 'author', 'en', 'Watch Cubs v Cardinals baseball livestream 9/14/2016', 2232, 1, 1473842389, 1473842389, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(36925, 36935, 'author', 'en', 'Watch Amur Khabarovsk - Neftekhimik Nizhnekamsk livestream September 22, 2016', 2240, 1, 1474500059, 1474500059, 0, 0, 0, 0, 0, 0),
(36929, 36939, 'author', 'en', 'Live streaming Admiral Vladivostok - Lada Togliatti KHL tv watch 9/22/2016', 2125, 1, 1474502287, 1474502287, 0, 0, 0, 0, 0, 0),
(36931, 36941, 'author', 'en', 'Live streaming Eyupspor - Gaziantepspor tv watch 22.09.2016', 2125, 1, 1474503371, 1474503371, 0, 0, 0, 0, 0, 0),
(36933, 36943, 'author', 'en', 'Watch Paolo Lorenzi v Alexandre Sidorenko Live', 2232, 1, 1474503645, 1474503645, 0, 0, 0, 0, 0, 0),
(36934, 36944, 'author', 'en', 'Watch Thailand U19 vs East Timor U19 Live 9/22/2016', 2235, 1, 1474503658, 1474503658, 0, 0, 0, 0, 0, 0),
(36935, 36945, 'author', 'en', 'Live streaming Admiral Vladivostok - Lada Togliatti KHL tv watch 22.09.2016', 2235, 1, 1474504205, 1474504205, 0, 0, 0, 0, 0, 0),
(36937, 36947, 'author', 'en', 'Watch Kunlun Red Star v Ak Bars hockey Live 22.09.2016', 2125, 1, 1474506449, 1474506449, 0, 0, 0, 0, 0, 0),
(36938, 36948, 'author', 'en', 'Millot - Jaziri Live Stream 9/22/2016', 2232, 1, 1474507053, 1474507053, 0, 0, 0, 0, 0, 0),
(36939, 36949, 'author', 'en', 'Troicki v Lajovic ATP St. Petersburg Live Stream 22.09.2016', 2232, 1, 1474507244, 1474507244, 0, 0, 0, 0, 0, 0),
(36941, 36951, 'author', 'en', 'Live streaming Malek Jaziri v Vincent Millot tv watch 22.09.2016', 2125, 1, 1474507612, 1474507612, 0, 0, 0, 0, 0, 0),
(36943, 36953, 'author', 'en', 'Stream online Ding Junhui v Mark Allen Shanghai Masters September 22, 2016', 2239, 1, 1474509748, 1474509748, 0, 0, 0, 0, 0, 0),
(36944, 36954, 'author', 'en', 'Watch Philippines vs Vietnam SEABA Championship Women Live 22.09.2016', 2125, 1, 1474511203, 1474511203, 0, 0, 0, 0, 0, 0),
(36945, 36955, 'author', 'en', 'South Korea U16 - Malaysia U16 AFC Championship U16 Live Stream 22.09.2016', 2125, 1, 1474513291, 1474513291, 0, 0, 0, 0, 0, 0),
(36949, 36959, 'author', 'en', 'Watch Prikamye Perm v Zenit Kazan Live 22.09.2016', 2239, 1, 1474517392, 1474517392, 0, 0, 0, 0, 0, 0),
(36950, 36960, 'author', 'en', 'Watch Kızılcabölükspor - Mersin live stream September 22, 2016', 2235, 1, 1474517500, 1474517500, 0, 0, 0, 0, 0, 0),
(36951, 36961, 'author', 'en', 'Live stream Kızılcabölükspor v Mersin soccer 22.09.2016', 2232, 1, 1474517540, 1474517540, 0, 0, 0, 0, 0, 0),
(36952, 36962, 'author', 'en', 'Live streaming Unirea Slobozia v SCM Craiova tv watch', 2239, 1, 1474518802, 1474518802, 0, 0, 0, 0, 0, 0),
(36954, 36964, 'author', 'en', 'Šiauliai vs Alytaus basketball Live Stream September 22, 2016', 2232, 1, 1474520223, 1474520223, 0, 0, 0, 0, 0, 0),
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(36959, 36969, 'author', 'en', 'Tarxien Rainbows - Hamrun Spartans Maltese Premier League Live Stream 22.09.2016', 2125, 1, 1474521000, 1474521000, 0, 0, 0, 0, 0, 0),
(36963, 36973, 'author', 'en', 'Watch Motor Zaporozhye vs HBC Nantes live stream 22.09.2016', 2232, 1, 1474521220, 1474521220, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
(38280, 38290, 'author', 'en', 'Watch Team EnVyUs vs Penta Sports esports Live', 2240, 1, 1474885238, 1474885238, 0, 0, 0, 0, 0, 0),
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(40842, 40852, 'author', 'en', 'Watch Santa Cruz Recife vs Palmeiras Brazilian Serie A Live 03.10.2016', 2236, 1, 1475500773, 1475500773, 0, 0, 0, 0, 0, 0),
(40843, 40853, 'author', 'en', 'NY Rangers - Philadelphia NHL Live Stream 03.10.2016', 2232, 1, 1475500919, 1475500919, 0, 0, 0, 0, 0, 0),
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(40851, 40861, 'author', 'en', 'Watch América de Cali - Valledupar Live 10/03/2016', 2239, 1, 1475503669, 1475503669, 0, 0, 0, 0, 0, 0),
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(40855, 40865, 'author', 'en', 'Watch stream Cartagena - Unión Magdalena Colombian Torneo Aguila October 03, 2016', 2236, 1, 1475504145, 1475504145, 0, 0, 0, 0, 0, 0),
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(40862, 40872, 'author', 'en', 'Watch Milwaukee vs Chicago Live October 03, 2016', 2239, 1, 1475505598, 1475505598, 0, 0, 0, 0, 0, 0),
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(40881, 40891, 'author', 'en', 'Live streaming Ottawa Senators v Winnipeg Jets NHL tv watch', 2240, 1, 1475507843, 1475507843, 0, 0, 0, 0, 0, 0),
(40882, 40892, 'author', 'en', 'Watch New Jersey - NY Islanders hockey Live October 03, 2016', 2240, 1, 1475508163, 1475508163, 0, 0, 0, 0, 0, 0),
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(40884, 40894, 'author', 'en', 'Watch Ottawa Senators - Winnipeg Jets live stream 03.10.2016', 2232, 1, 1475508186, 1475508186, 0, 0, 0, 0, 0, 0),
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(40888, 40898, 'author', 'en', 'Watch Milwaukee Bucks v Chicago Bulls Live', 2232, 1, 1475508655, 1475508655, 0, 0, 0, 0, 0, 0),
(40891, 40901, 'author', 'en', 'Watch Utah vs Portland basketball live streaming', 2238, 1, 1475509097, 1475509097, 0, 0, 0, 0, 0, 0),
(40892, 40902, 'author', 'en', 'Watch Denver vs Toronto NBA Live 03.10.2016', 2235, 1, 1475509294, 1475509294, 0, 0, 0, 0, 0, 0),
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(40894, 40904, 'author', 'en', 'Watch San Antonio vs Phoenix live streaming October 03, 2016', 2235, 1, 1475509526, 1475509526, 0, 0, 0, 0, 0, 0),
(40895, 40905, 'author', 'en', 'Watch Utah Jazz - Portland Trail Blazers basketball Live October 03, 2016', 2240, 1, 1475509575, 1475509575, 0, 0, 0, 0, 0, 0),
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(40901, 40911, 'author', 'en', 'Watch Sabine Lisicki - Elina Svitolina WTA Beijing live streaming 10/03/2016', 2240, 1, 1475510648, 1475510648, 0, 0, 0, 0, 0, 0),
(40902, 40912, 'author', 'en', 'Watch NY Giants vs Minnesota Live', 2232, 1, 1475510678, 1475510678, 0, 0, 0, 0, 0, 0),
(40903, 40913, 'author', 'en', 'Watch Magic - Grizzlies live stream 03.10.2016', 2240, 1, 1475510731, 1475510731, 0, 0, 0, 0, 0, 0),
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(40908, 40918, 'author', 'en', 'Live streaming Makarova v Radwanska tennis tv watch October 03, 2016', 2232, 1, 1475511004, 1475511004, 0, 0, 0, 0, 0, 0),
(40909, 40919, 'author', 'en', 'Watch Milwaukee vs Chicago NBA Live', 2125, 1, 1475511193, 1475511193, 0, 0, 0, 0, 0, 0),
(40914, 40924, 'author', 'en', 'Watch Wang - Kvitova Live 03.10.2016', 2235, 1, 1475512123, 1475512123, 0, 0, 0, 0, 0, 0),
(40915, 40925, 'author', 'en', 'Watch Kuznetsova vs Doi WTA Beijing Live 03.10.2016', 2125, 1, 1475512169, 1475512169, 0, 0, 0, 0, 0, 0),
(40917, 40927, 'author', 'en', 'Watch San Antonio v Phoenix Live', 2125, 1, 1475512475, 1475512475, 0, 0, 0, 0, 0, 0),
(40919, 40929, 'author', 'en', 'Watch Denver v Toronto live stream 03.10.2016', 2239, 1, 1475513292, 1475513292, 0, 0, 0, 0, 0, 0),
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(40922, 40932, 'author', 'en', 'Spurs - Suns basketball Live Stream', 2239, 1, 1475514156, 1475514156, 0, 0, 0, 0, 0, 0),
(40923, 40933, 'author', 'en', 'Watch Online Stream Makarova vs Radwanska  03.10.2016', 2236, 1, 1475514283, 1475514283, 0, 0, 0, 0, 0, 0),
(40924, 40934, 'author', 'en', 'Watch Pella vs Jaziri tennis Live 03.10.2016', 2240, 1, 1475514330, 1475514330, 0, 0, 0, 0, 0, 0),
(40926, 40936, 'author', 'en', 'Watch Andy Murray vs Andreas Seppi ATP Beijing live stream', 2239, 1, 1475514653, 1475514653, 0, 0, 0, 0, 0, 0),
(40929, 40939, 'author', 'en', 'Watch Harrison - Kyrgios tennis live streaming 10/03/2016', 2232, 1, 1475515234, 1475515234, 0, 0, 0, 0, 0, 0),
(40930, 40940, 'author', 'en', 'Watch Elina Svitolina vs Sabine Lisicki Live 03.10.2016', 2238, 1, 1475515460, 1475515460, 0, 0, 0, 0, 0, 0),
(40931, 40941, 'author', 'en', 'Watch Querrey vs Gasquet live streaming 10/03/2016', 2125, 1, 1475515559, 1475515559, 0, 0, 0, 0, 0, 0),
(40933, 40943, 'author', 'en', 'Zhang vs Riske WTA Beijing Live Stream 10/03/2016', 2238, 1, 1475515854, 1475515854, 0, 0, 0, 0, 0, 0),
(40934, 40944, 'author', 'en', 'Marcos Baghdatis v Marcel Granollers-Pujol ATP Tokyo Live Stream October 03, 2016', 2239, 1, 1475515928, 1475515928, 0, 0, 0, 0, 0, 0),
(40935, 40945, 'author', 'en', 'Watch Petra Kvitova - Yafan Wang Live 10/03/2016', 2232, 1, 1475516172, 1475516172, 0, 0, 0, 0, 0, 0),
(40938, 40948, 'author', 'en', 'Live streaming James Duckworth vs Feliciano Lopez tv watch October 03, 2016', 2239, 1, 1475516301, 1475516301, 0, 0, 0, 0, 0, 0),
(40942, 40952, 'author', 'en', 'Lorenzi - Nadal ATP Beijing Live Stream October 03, 2016', 2232, 1, 1475516726, 1475516726, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(40962, 40972, 'author', 'en', 'Cornet vs Cibulkova tennis Live Stream 03.10.2016', 2125, 1, 1475520458, 1475520458, 0, 0, 0, 0, 0, 0),
(40963, 40973, 'author', 'en', 'Watch Goffin v Nishioka tennis live stream October 03, 2016', 2240, 1, 1475520497, 1475520497, 0, 0, 0, 0, 0, 0),
(40964, 40974, 'author', 'en', 'Watch live Spurs - Suns basketball October 03, 2016', 2240, 1, 1475521321, 1475521321, 0, 0, 0, 0, 0, 0),
(40966, 40976, 'author', 'en', 'Watch Riske - Zhang tennis live streaming October 03, 2016', 2236, 1, 1475521686, 1475521686, 0, 0, 0, 0, 0, 0),
(40968, 40978, 'author', 'en', 'Watch Taylor Harry Fritz vs Juan Monaco tennis live stream October 03, 2016', 2125, 1, 1475521724, 1475521724, 0, 0, 0, 0, 0, 0),
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(40972, 40982, 'author', 'en', 'Watch Delbonis v Karlovic Live', 2240, 1, 1475522008, 1475522008, 0, 0, 0, 0, 0, 0),
(40973, 40983, 'author', 'en', 'Zhang - Sock Live Stream 03.10.2016', 2238, 1, 1475522183, 1475522183, 0, 0, 0, 0, 0, 0),
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(40975, 40985, 'author', 'en', 'Karolina Pliskova - Darya Kasatkina WTA Beijing Live Stream 03.10.2016', 2239, 1, 1475522371, 1475522371, 0, 0, 0, 0, 0, 0),
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(40980, 40990, 'author', 'en', 'Watch Marcel Granollers-Pujol vs Marcos Baghdatis Live 03.10.2016', 2238, 1, 1475522825, 1475522825, 0, 0, 0, 0, 0, 0),
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(40984, 40994, 'author', 'en', 'Watch Marcel Granollers-Pujol - Marcos Baghdatis tennis Live 04.10.2016', 2239, 1, 1475523052, 1475523052, 0, 0, 0, 0, 0, 0),
(40985, 40995, 'author', 'en', 'Live streaming Caroline Wozniacki v Roberta Vinci tv watch', 2240, 1, 1475523061, 1475523061, 0, 0, 0, 0, 0, 0),
(40986, 40996, 'author', 'en', 'Live streaming Taro Daniel v Janko Tipsarevic ATP Tokyo tv watch October 03, 2016', 2238, 1, 1475523147, 1475523147, 0, 0, 0, 0, 0, 0),
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(40994, 41004, 'author', 'en', 'Live streaming Kohlschreiber - Simon ATP Tokyo tv watch', 2239, 1, 1475524958, 1475524958, 0, 0, 0, 0, 0, 0),
(40997, 41007, 'author', 'en', 'Live streaming Kravchuk - Kuznetsov tv watch October 04, 2016', 2236, 1, 1475525202, 1475525202, 0, 0, 0, 0, 0, 0),
(40998, 41008, 'author', 'en', 'Live streaming Federico Delbonis v Ivo Karlovic tennis tv watch', 2238, 1, 1475525371, 1475525371, 0, 0, 0, 0, 0, 0),
(40999, 41009, 'author', 'en', 'Watch Kuznetsova v Doi tennis Live 10/03/2016', 2232, 1, 1475525701, 1475525701, 0, 0, 0, 0, 0, 0),
(41000, 41010, 'author', 'en', 'Guido Pella - Malek Jaziri ATP Beijing Live Stream', 2240, 1, 1475525848, 1475525848, 0, 0, 0, 0, 0, 0),
(41002, 41012, 'author', 'en', 'Sugita - Monfils Live Stream 10/04/2016', 2232, 1, 1475526062, 1475526062, 0, 0, 0, 0, 0, 0),
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(41010, 41020, 'author', 'en', 'Watch Online Stream Alexander Zverev v Dominic Thiem tennis October 04, 2016', 2235, 1, 1475527251, 1475527251, 0, 0, 0, 0, 0, 0),
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(41013, 41023, 'author', 'en', 'Live streaming Steve Johnson - Grigor Dimitrov ATP Beijing tv watch October 03, 2016', 2235, 1, 1475528459, 1475528459, 0, 0, 0, 0, 0, 0),
(41014, 41024, 'author', 'en', 'Petra Kvitova v Yafan Wang WTA Beijing Live Stream', 2125, 1, 1475528623, 1475528623, 0, 0, 0, 0, 0, 0),
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(41026, 41036, 'author', 'en', 'Live streaming Guido Pella - Malek Jaziri tv watch October 04, 2016', 2235, 1, 1475533116, 1475533116, 0, 0, 0, 0, 0, 0),
(41027, 41037, 'author', 'en', 'Live streaming Carreno Busta vs Tomic tv watch October 04, 2016', 2232, 1, 1475533407, 1475533407, 0, 0, 0, 0, 0, 0),
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(41561, 41571, 'conference_paper', 'en', 'Genetic diversity of Helicobacter pylori isolates: the impact on disease outcomes in different populations', 2414, 1, 1475647754, 1475711244, 0, 0, 0, 0, 0, 0),
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(41842, 41852, 'author', 'en', 'Japan - Iraq Live Stream 06.10.2016', 2236, 1, 1475719199, 1475719199, 0, 0, 0, 0, 0, 0),
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(41849, 41859, 'author', 'en', 'China - Syria soccer Live Stream 06.10.2016', 2232, 1, 1475720706, 1475720706, 0, 0, 0, 0, 0, 0),
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(41851, 41861, 'author', 'en', 'Kyrgyzstan - Lebanon soccer Live Stream 06.10.2016', 2239, 1, 1475720892, 1475720892, 0, 0, 0, 0, 0, 0),
(41852, 41862, 'author', 'en', 'Watch Malek Jaziri - Milos Raonic ATP Beijing live stream 06.10.2016', 2236, 1, 1475721081, 1475721081, 0, 0, 0, 0, 0, 0),
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(41858, 41868, 'author', 'en', 'Watch Chennaiyin v Delhi Dynamos Indian ISL live stream October 06, 2016', 2125, 1, 1475722994, 1475722994, 0, 0, 0, 0, 0, 0),
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(41860, 41870, 'author', 'en', 'Watch Fitriani - Ongbamrungphan Grand Prix Thailand Open Women live stream October 06, 2016', 2235, 1, 1475723637, 1475723637, 0, 0, 0, 0, 0, 0),
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(41871, 41881, 'author', 'en', 'Watch China vs Syria live streaming October 06, 2016', 2125, 1, 1475724848, 1475724848, 0, 0, 0, 0, 0, 0),
(41874, 41884, 'author', 'en', 'Watch Online Stream Dzumhur - Majchrzak Mohammedia Challenger October 06, 2016', 2239, 1, 1475725424, 1475725424, 0, 0, 0, 0, 0, 0),
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(41920, 41930, 'author', 'en', 'Watch Poland U21 v Ukraine U20 Friendly live streaming 06.10.2016', 2240, 1, 1475734040, 1475734040, 0, 0, 0, 0, 0, 0),
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(41938, 41948, 'author', 'en', 'SaiPa - TPS Finnish SM-liiga Live Stream', 2125, 1, 1475735830, 1475735830, 0, 0, 0, 0, 0, 0),
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(41947, 41957, 'author', 'en', 'KK Zadar vs Crvena Zvezda Live Stream 10/06/2016', 2236, 1, 1475738160, 1475738160, 0, 0, 0, 0, 0, 0),
(41948, 41958, 'author', 'en', 'Dinamo Riga - Lokomotiv KHL Live Stream 06.10.2016', 2238, 1, 1475738185, 1475738185, 0, 0, 0, 0, 0, 0),
(41949, 41959, 'author', 'en', 'Watch Italy v Spain soccer live streaming October 06, 2016', 2235, 1, 1475738323, 1475738323, 0, 0, 0, 0, 0, 0),
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(41958, 41968, 'author', 'en', 'Hungary U21 vs Portugal U21 Live Stream 06.10.2016', 2125, 1, 1475738949, 1475738949, 0, 0, 0, 0, 0, 0);
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(41979, 41989, 'author', 'en', 'Live streaming France U21 vs Georgia U21 Friendly 10/06/2016', 2240, 1, 1475741201, 1475741201, 0, 0, 0, 0, 0, 0),
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(43469, 43479, 'author', 'en', 'Netherlands - France soccer Live Stream 10.10.2016', 2125, 1, 1476087651, 1476087651, 0, 0, 0, 0, 0, 0),
(43471, 43481, 'author', 'en', 'England U20 vs USA U20 Friendly Live Stream', 2125, 1, 1476087900, 1476087900, 0, 0, 0, 0, 0, 0);
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(44382, 44392, 'author', 'en', 'Denver Nuggets v Minnesota Timberwolves basketball Live Stream 12.10.2016', 2232, 1, 1476288600, 1476288600, 0, 0, 0, 0, 0, 0),
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(44393, 44403, 'author', 'en', 'Wozniacki vs Watson tennis Live Stream 10/12/2016', 2238, 1, 1476289332, 1476289332, 0, 0, 0, 0, 0, 0),
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(44397, 44407, 'author', 'en', 'Roberto Bautista-Agut - Viktor Troicki Live Stream October 12, 2016', 2235, 1, 1476289829, 1476289829, 0, 0, 0, 0, 0, 0),
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(44480, 44490, 'author', 'en', 'Goffin - Monfils tennis Live Stream 10/13/2016', 2232, 1, 1476316776, 1476316776, 0, 0, 0, 0, 0, 0),
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(44499, 44509, 'author', 'en', 'Live streaming Pakistan vs West Indies Test Series 13.10.2016', 2238, 1, 1476325394, 1476325394, 0, 0, 0, 0, 0, 0);
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(50526, 50536, 'author', 'en', 'Watch Golden State v Phoenix live streaming October 30, 2016', 2236, 1, 1477830137, 1477830137, 0, 0, 0, 0, 0, 0),
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(50583, 50593, 'author', 'en', 'Watch Puebla - León soccer Live', 2239, 1, 1477837751, 1477837751, 0, 0, 0, 0, 0, 0),
(50585, 50595, 'author', 'en', 'Watch Levante - Getafe Spanish Segunda Division Live October 30, 2016', 2236, 1, 1477837950, 1477837950, 0, 0, 0, 0, 0, 0),
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(50590, 50600, 'author', 'en', 'Watch Philadelphia Eagles vs Dallas Cowboys football live streaming October 30, 2016', 2238, 1, 1477840223, 1477840223, 0, 0, 0, 0, 0, 0),
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(50593, 50603, 'author', 'en', 'Live streaming Philadelphia - Dallas NFL tv watch October 30, 2016', 2125, 1, 1477840466, 1477840466, 0, 0, 0, 0, 0, 0),
(50594, 50604, 'author', 'en', 'Live streaming Barton - Nolan tv watch', 2236, 1, 1477840471, 1477840471, 0, 0, 0, 0, 0, 0),
(50596, 50606, 'author', 'en', 'Live streaming Washington Wizards vs Memphis Grizzlies tv watch October 30, 2016', 2240, 1, 1477840816, 1477840816, 0, 0, 0, 0, 0, 0),
(50598, 50608, 'author', 'en', 'Live streaming Dallas Mavericks v Houston Rockets basketball tv watch', 2240, 1, 1477840936, 1477840936, 0, 0, 0, 0, 0, 0),
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(50600, 50610, 'author', 'en', 'Watch Deportes Temuco v Universidad de Concepción livestream 30.10.2016', 2125, 1, 1477841303, 1477841303, 0, 0, 0, 0, 0, 0),
(50602, 50612, 'author', 'en', 'Watch Deportivo Carchá v Marquense Live 10/30/2016', 2125, 1, 1477841538, 1477841538, 0, 0, 0, 0, 0, 0),
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(50605, 50615, 'author', 'en', 'San Antonio Spurs - Miami Heat NBA Live Stream', 2236, 1, 1477841604, 1477841604, 0, 0, 0, 0, 0, 0),
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(50613, 50623, 'author', 'en', 'Watch Dallas Mavericks vs Houston Rockets live streaming', 2238, 1, 1477843706, 1477843706, 0, 0, 0, 0, 0, 0),
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(50616, 50626, 'author', 'en', 'Live streaming Washington - Memphis basketball tv watch 30.10.2016', 2239, 1, 1477843911, 1477843911, 0, 0, 0, 0, 0, 0),
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(50624, 50634, 'author', 'en', 'Watch live Puebla - León Mexican Primera Division', 2235, 1, 1477845116, 1477845116, 0, 0, 0, 0, 0, 0),
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(50627, 50637, 'author', 'en', 'Seattle Thunderbirds v Regina Pats WHL Live Stream 10/30/2016', 2239, 1, 1477845470, 1477845470, 0, 0, 0, 0, 0, 0),
(50628, 50638, 'author', 'en', 'Live streaming Nacional v River Plate soccer tv watch 30.10.2016', 2239, 1, 1477845805, 1477845805, 0, 0, 0, 0, 0, 0),
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(50630, 50640, 'author', 'en', 'LA Lakers - Oklahoma City Live Stream October 30, 2016', 2232, 1, 1477845886, 1477845886, 0, 0, 0, 0, 0, 0),
(50631, 50641, 'author', 'en', 'Sol de América v General Díaz Paraguayan Primera Division Live Stream', 2240, 1, 1477847205, 1477847205, 0, 0, 0, 0, 0, 0),
(50632, 50642, 'author', 'en', 'Watch Seattle Sounders - FC Dallas MLS Live', 2239, 1, 1477847441, 1477847441, 0, 0, 0, 0, 0, 0),
(50634, 50644, 'author', 'en', 'Watch Eagles v Cowboys football livestream October 30, 2016', 2239, 1, 1477847541, 1477847541, 0, 0, 0, 0, 0, 0),
(50638, 50648, 'author', 'en', 'Ottawa vs Edmonton Live Stream', 2240, 1, 1477848781, 1477848781, 0, 0, 0, 0, 0, 0),
(50639, 50649, 'author', 'en', 'Watch Kohlschreiber - Sock livestream October 31, 2016', 2236, 1, 1477864999, 1477864999, 0, 0, 0, 0, 0, 0),
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(50891, 50902, 'author', 'en', 'Ludogorets U19 vs Arsenal U19 soccer Live Stream 01.11.2016', 2125, 1, 1477973889, 1477973889, 0, 0, 0, 0, 0, 0),
(50892, 50903, 'author', 'en', 'Duckworth v Van Peperzeel Live Stream 01.11.2016', 2240, 1, 1477973993, 1477973993, 0, 0, 0, 0, 0, 0),
(50893, 50904, 'author', 'en', 'Watch Manchester City U19 - Barcelona U19 live streaming 11/01/2016', 2238, 1, 1477974011, 1477974011, 0, 0, 0, 0, 0, 0),
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INSERT INTO `node` (`nid`, `vid`, `type`, `language`, `title`, `uid`, `status`, `created`, `changed`, `comment`, `promote`, `moderate`, `sticky`, `tnid`, `translate`) VALUES
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(52469, 52481, 'author', 'en', 'Watch Izarra - Caudal Deportivo live stream November 06, 2016', 2236, 1, 1478419272, 1478419272, 0, 0, 0, 0, 0, 0),
(52470, 52482, 'author', 'en', 'Live streaming Liverpool vs Watford tv watch November 06, 2016', 2236, 1, 1478419618, 1478419618, 0, 0, 0, 0, 0, 0),
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(52473, 52485, 'author', 'en', 'Malmö - Hammarby Swedish Allsvenskan Live Stream 06.11.2016', 2240, 1, 1478420027, 1478420027, 0, 0, 0, 0, 0, 0),
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(52476, 52488, 'author', 'en', 'Watch Konyaspor vs Kasımpaşa Turkish Super League live streaming November 06, 2016', 2236, 1, 1478420155, 1478420155, 0, 0, 0, 0, 0, 0),
(52477, 52489, 'author', 'en', 'Watch København - SønderjyskE soccer Live 06.11.2016', 2232, 1, 1478420465, 1478420465, 0, 0, 0, 0, 0, 0),
(52478, 52490, 'author', 'en', 'Stream online Liverpool - Watford English Premier League 06.11.2016', 2240, 1, 1478420471, 1478420471, 0, 0, 0, 0, 0, 0),
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(52484, 52496, 'author', 'en', 'Watch Sassuolo - Atalanta Live 06.11.2016', 2235, 1, 1478421414, 1478421414, 0, 0, 0, 0, 0, 0),
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(52487, 52499, 'author', 'en', 'Finland - Sweden Euro Hockey Tour - Karjala Cup Live Stream November 06, 2016', 2236, 1, 1478421875, 1478421875, 0, 0, 0, 0, 0, 0),
(52488, 52500, 'author', 'en', 'Watch Reggiana - Cantù basketball live streaming 06.11.2016', 2125, 1, 1478421884, 1478421884, 0, 0, 0, 0, 0, 0),
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(52494, 52506, 'author', 'en', 'Live streaming Panionios - PAOK soccer tv watch', 2238, 1, 1478422960, 1478422960, 0, 0, 0, 0, 0, 0),
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(53304, 53316, 'author', 'en', 'Watch KK Budućnost - Bayern München Eurocup Live 09.11.2016', 2240, 1, 1478684941, 1478684941, 0, 0, 0, 0, 0, 0),
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(53409, 53421, 'author', 'en', 'Brooklyn - New York Live Stream 09.11.2016', 2239, 1, 1478704518, 1478704518, 0, 0, 0, 0, 0, 0),
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(53433, 53445, 'author', 'en', 'Watch Ottawa Senators vs Buffalo Sabres Live November 09, 2016', 2232, 1, 1478707657, 1478707657, 0, 0, 0, 0, 0, 0),
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(53931, 53943, 'author', 'en', 'Live streaming Armenia vs Montenegro FIFA World Cup tv watch November 11, 2016', 2235, 1, 1478857160, 1478857160, 0, 0, 0, 0, 0, 0),
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(53995, 54007, 'author', 'en', 'Watch Montana State Bobcats v Washington State Cougars NCAA College Basketball Live 11.11.2016', 2236, 1, 1478870074, 1478870074, 0, 0, 0, 0, 0, 0),
(53997, 54009, 'author', 'en', 'Watch Trinidad & Tobago - Costa Rica soccer Live November 11, 2016', 2238, 1, 1478870334, 1478870334, 0, 0, 0, 0, 0, 0),
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(54002, 54014, 'author', 'en', 'England v Scotland FIFA World Cup Live Stream November 11, 2016', 2238, 1, 1478872026, 1478872026, 0, 0, 0, 0, 0, 0),
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(54009, 54021, 'author', 'en', 'Live streaming New York Knicks - Boston Celtics NBA tv watch 11.11.2016', 2239, 1, 1478873525, 1478873525, 0, 0, 0, 0, 0, 0),
(54010, 54022, 'author', 'en', 'Live streaming Venezuela U20 - Peru U20 tv watch November 11, 2016', 2240, 1, 1478873669, 1478873669, 0, 0, 0, 0, 0, 0),
(54011, 54023, 'author', 'en', 'Watch Venezuela U20 - Peru U20 soccer live stream', 2239, 1, 1478873705, 1478873705, 0, 0, 0, 0, 0, 0),
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(54014, 54026, 'author', 'en', 'Watch Mount St. Mary''s vs West Virginia NCAA College Basketball Live November 11, 2016', 2239, 1, 1478874059, 1478874059, 0, 0, 0, 0, 0, 0),
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(68557, 68572, 'author', 'en', 'Bactroban | Buy Topical', 2547, 1, 1487140532, 1487140532, 0, 0, 0, 0, 0, 0),
(68558, 68573, 'author', 'en', 'Diamox | Buy In Lima', 2547, 1, 1487140551, 1487140551, 0, 0, 0, 0, 0, 0),
(68559, 68574, 'author', 'en', 'Tinidazole | Where To Buy 500Mg', 2547, 1, 1487140565, 1487140565, 0, 0, 0, 0, 0, 0),
(68560, 68575, 'author', 'en', 'Hyzaar | Order 100', 2551, 1, 1487143401, 1487143401, 0, 0, 0, 0, 0, 0),
(68561, 68576, 'author', 'en', 'Provera | Can You Order Online', 2551, 1, 1487143432, 1487143432, 0, 0, 0, 0, 0, 0),
(68562, 68577, 'author', 'en', 'Biaxin | Purchase Antibiotic', 2549, 1, 1487161946, 1487161946, 0, 0, 0, 0, 0, 0),
(68563, 68578, 'author', 'en', 'Lamictal | Purchase For Depression', 2549, 1, 1487161961, 1487161961, 0, 0, 0, 0, 0, 0),
(68564, 68579, 'author', 'en', 'Naltrexone | Buy In Spain', 2549, 1, 1487200546, 1487200546, 0, 0, 0, 0, 0, 0),
(68565, 68580, 'author', 'en', 'Naltrexone | Buy In The Uk', 2549, 1, 1487200579, 1487200579, 0, 0, 0, 0, 0, 0),
(68566, 68581, 'author', 'en', 'ประกันภัยรถยนต์', 2385, 1, 1487459628, 1487459628, 0, 0, 0, 0, 0, 0),
(68567, 68582, 'author', 'en', 'Nurul ''Izzah A G', 2555, 1, 1487467558, 1487467558, 0, 0, 0, 0, 0, 0),
(68568, 68583, 'author', 'en', 'Chowdhury S R', 2555, 1, 1487468041, 1487468041, 0, 0, 0, 0, 0, 0),
(68569, 68584, 'author', 'en', 'Aminuddin B S', 2555, 1, 1487468375, 1487468375, 0, 0, 0, 0, 0, 0),
(68570, 68585, 'author', 'en', 'Ruszymah B H I', 2555, 1, 1487468600, 1487468600, 0, 0, 0, 0, 0, 0),
(68571, 68586, 'conference_paper', 'en', 'DERMAL FIBROBLASTS CONDITIONED MEDIUM FACILITATES IN VITRO RE-EPITHELIALIZATION IRRESPECTIVE OF DONOR AGE', 2555, 1, 1487469681, 1487469681, 0, 0, 0, 0, 0, 0),
(68572, 68587, 'author', 'en', 'Naltrexone | Buy Low Dose India', 2551, 1, 1487474026, 1487474026, 0, 0, 0, 0, 0, 0),
(68573, 68588, 'author', 'en', 'ประกันบิ๊กไบค์', 2385, 1, 1487545867, 1487545867, 0, 0, 0, 0, 0, 0);

-- --------------------------------------------------------

--
-- Table structure for table `nodequeue_nodes`
--

CREATE TABLE IF NOT EXISTS `nodequeue_nodes` (
  `qid` int(10) unsigned NOT NULL,
  `sqid` int(10) unsigned NOT NULL,
  `nid` int(10) unsigned DEFAULT NULL,
  `position` int(10) unsigned DEFAULT NULL,
  `timestamp` int(10) unsigned NOT NULL DEFAULT '0',
  KEY `nodequeue_nodes_sqid_idx` (`sqid`,`position`),
  KEY `nodequeue_subqueue_nid_idx` (`nid`),
  KEY `nodequeue_nodes_qid_nid_idx` (`qid`,`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

-- --------------------------------------------------------

--
-- Table structure for table `nodequeue_queue`
--

CREATE TABLE IF NOT EXISTS `nodequeue_queue` (
  `qid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `title` varchar(255) NOT NULL,
  `subqueue_title` varchar(255) NOT NULL,
  `size` int(11) DEFAULT '0',
  `link` varchar(40) DEFAULT NULL,
  `link_remove` varchar(40) DEFAULT NULL,
  `owner` varchar(255) DEFAULT NULL,
  `show_in_ui` tinyint(4) DEFAULT '1',
  `show_in_tab` tinyint(4) DEFAULT '1',
  `show_in_links` tinyint(4) DEFAULT '1',
  `reference` varchar(255) DEFAULT '0',
  `reverse` tinyint(4) DEFAULT NULL,
  `i18n` tinyint(4) DEFAULT '1',
  PRIMARY KEY (`qid`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=2 ;

--
-- Dumping data for table `nodequeue_queue`
--

INSERT INTO `nodequeue_queue` (`qid`, `title`, `subqueue_title`, `size`, `link`, `link_remove`, `owner`, `show_in_ui`, `show_in_tab`, `show_in_links`, `reference`, `reverse`, `i18n`) VALUES
(1, 'Submission', '', 0, '', '', 'nodequeue', 1, 1, 0, '0', 0, 0);

-- --------------------------------------------------------

--
-- Table structure for table `nodequeue_roles`
--

CREATE TABLE IF NOT EXISTS `nodequeue_roles` (
  `qid` bigint(20) unsigned NOT NULL,
  `rid` bigint(20) unsigned NOT NULL,
  KEY `nodequeue_roles_qid_idx` (`qid`),
  KEY `nodequeue_roles_rid_idx` (`rid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `nodequeue_roles`
--

INSERT INTO `nodequeue_roles` (`qid`, `rid`) VALUES
(1, 3);

-- --------------------------------------------------------

--
-- Table structure for table `nodequeue_subqueue`
--

CREATE TABLE IF NOT EXISTS `nodequeue_subqueue` (
  `sqid` int(10) unsigned NOT NULL AUTO_INCREMENT,
  `qid` int(10) unsigned NOT NULL,
  `reference` varchar(255) DEFAULT '0',
  `title` varchar(255) DEFAULT '',
  PRIMARY KEY (`sqid`),
  KEY `nodequeue_subqueue_qid_idx` (`qid`),
  KEY `nodequeue_subqueue_reference_idx` (`reference`),
  KEY `nodequeue_subqueue_title_idx` (`title`)
) ENGINE=InnoDB  DEFAULT CHARSET=utf8 AUTO_INCREMENT=2 ;

--
-- Dumping data for table `nodequeue_subqueue`
--

INSERT INTO `nodequeue_subqueue` (`sqid`, `qid`, `reference`, `title`) VALUES
(1, 1, '1', 'Submission');

-- --------------------------------------------------------

--
-- Table structure for table `nodequeue_types`
--

CREATE TABLE IF NOT EXISTS `nodequeue_types` (
  `qid` bigint(20) unsigned NOT NULL,
  `type` varchar(255) DEFAULT NULL,
  KEY `nodequeue_types_qid_idx` (`qid`),
  KEY `nodequeue_types_type_idx` (`type`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `nodequeue_types`
--

INSERT INTO `nodequeue_types` (`qid`, `type`) VALUES
(1, 'conference_paper');

-- --------------------------------------------------------

--
-- Table structure for table `noderelationships_settings`
--

CREATE TABLE IF NOT EXISTS `noderelationships_settings` (
  `type_name` varchar(32) NOT NULL DEFAULT '',
  `relation_type` varchar(10) NOT NULL DEFAULT '',
  `related_type` varchar(32) NOT NULL DEFAULT '',
  `field_name` varchar(32) NOT NULL DEFAULT '',
  `settings` mediumtext NOT NULL,
  `export_key` varchar(64) NOT NULL DEFAULT '',
  PRIMARY KEY (`type_name`,`relation_type`,`related_type`,`field_name`),
  KEY `type_field_relation` (`type_name`,`field_name`,`relation_type`),
  KEY `related_field_relation` (`related_type`,`field_name`,`relation_type`),
  KEY `field_name` (`field_name`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `noderelationships_settings`
--

INSERT INTO `noderelationships_settings` (`type_name`, `relation_type`, `related_type`, `field_name`, `settings`, `export_key`) VALUES
('article', 'noderef', '', 'field_mid', 'a:2:{s:25:"search_and_reference_view";s:37:"noderelationships2_noderef:page_table";s:18:"view_in_new_window";s:9:"field_mid";}', 'article__noderef____field_mid'),
('article', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:37:"noderelationships2_noderef:page_table";s:20:"create_and_reference";s:15:"field_op_author";}', 'article__noderef____field_op_author'),
('article', 'noderef', '', 'field_op_related_mmedia', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:23:"field_op_related_mmedia";}', 'article__noderef____field_op_related_mmedia'),
('article', 'noderef', '', 'field_related_content', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:21:"field_related_content";}', 'article__noderef____field_related_content'),
('audio', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:36:"noderelationships_noderef:page_table";s:20:"create_and_reference";s:15:"field_op_author";}', 'audio__noderef____field_op_author'),
('blog', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:15:"field_op_author";}', 'blog__noderef____field_op_author'),
('conference_paper', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:37:"noderelationships2_noderef:page_table";s:20:"create_and_reference";s:15:"field_op_author";}', 'conference_paper__noderef____field_op_author'),
('package', 'noderef', '', 'field_center_related', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:20:"field_center_related";}', 'package__noderef____field_center_related'),
('package', 'noderef', '', 'field_left_related', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:18:"field_left_related";}', 'package__noderef____field_left_related'),
('package', 'noderef', '', 'field_right_related', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:19:"field_right_related";}', 'package__noderef____field_right_related'),
('resource', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:35:"noderelationships_noderef:page_grid";s:20:"create_and_reference";s:15:"field_op_author";}', 'resource__noderef____field_op_author'),
('slideshow', 'noderef', '', 'field_op_author', 'a:3:{s:25:"search_and_reference_view";s:36:"noderelationships_noderef:page_table";s:20:"create_and_reference";s:15:"field_op_author";s:23:"translate_and_reference";s:15:"field_op_author";}', 'slideshow__noderef____field_op_author'),
('slideshow', 'noderef', '', 'field_op_slideshow_images', 'a:3:{s:25:"search_and_reference_view";s:36:"noderelationships_noderef:page_table";s:20:"create_and_reference";s:25:"field_op_slideshow_images";s:23:"translate_and_reference";s:25:"field_op_slideshow_images";}', 'slideshow__noderef____field_op_slideshow_images'),
('video', 'noderef', '', 'field_op_author', 'a:2:{s:25:"search_and_reference_view";s:36:"noderelationships_noderef:page_table";s:20:"create_and_reference";s:15:"field_op_author";}', 'video__noderef____field_op_author');

-- --------------------------------------------------------

--
-- Table structure for table `nodewords`
--

CREATE TABLE IF NOT EXISTS `nodewords` (
  `mtid` int(11) NOT NULL AUTO_INCREMENT,
  `type` smallint(5) unsigned NOT NULL DEFAULT '0',
  `id` int(10) unsigned NOT NULL DEFAULT '0',
  `name` varchar(32) NOT NULL DEFAULT '',
  `content` longtext NOT NULL,
  PRIMARY KEY (`mtid`),
  UNIQUE KEY `nodewords_type_id_name` (`type`,`id`,`name`),
  KEY `nodewords_name` (`name`(6)),
  KEY `nodewords_type_id` (`type`,`id`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8 AUTO_INCREMENT=1 ;

-- --------------------------------------------------------

--
-- Table structure for table `node_access`
--

CREATE TABLE IF NOT EXISTS `node_access` (
  `nid` int(10) unsigned NOT NULL DEFAULT '0',
  `gid` int(10) unsigned NOT NULL DEFAULT '0',
  `realm` varchar(255) NOT NULL DEFAULT '',
  `grant_view` tinyint(3) unsigned NOT NULL DEFAULT '0',
  `grant_update` tinyint(3) unsigned NOT NULL DEFAULT '0',
  `grant_delete` tinyint(3) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`nid`,`gid`,`realm`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `node_access`
--

INSERT INTO `node_access` (`nid`, `gid`, `realm`, `grant_view`, `grant_update`, `grant_delete`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `node_comment_statistics`
--

CREATE TABLE IF NOT EXISTS `node_comment_statistics` (
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  `comment_count` int(10) unsigned NOT NULL DEFAULT '0',
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) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `node_comment_statistics`
--

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-- --------------------------------------------------------

--
-- Table structure for table `node_counter`
--

CREATE TABLE IF NOT EXISTS `node_counter` (
  `nid` int(11) NOT NULL DEFAULT '0',
  `totalcount` bigint(20) unsigned NOT NULL DEFAULT '0',
  `daycount` mediumint(8) unsigned NOT NULL DEFAULT '0',
  `timestamp` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`nid`)
) ENGINE=InnoDB DEFAULT CHARSET=utf8;

--
-- Dumping data for table `node_counter`
--

INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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(56232, 16, 16, 1486557210),
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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(65702, 1, 1, 1480706391);
INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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INSERT INTO `node_counter` (`nid`, `totalcount`, `daycount`, `timestamp`) VALUES
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-- --------------------------------------------------------

--
-- Table structure for table `node_import_status`
--

CREATE TABLE IF NOT EXISTS `node_import_status` (
  `taskid` int(10) unsigned NOT NULL DEFAULT '0',
  `file_offset` int(10) unsigned NOT NULL DEFAULT '0',
  `errors` longtext NOT NULL,
  `objid` int(10) unsigned NOT NULL DEFAULT '0',
  `status` int(10) unsigned NOT NULL DEFAULT '0',
  PRIMARY KEY (`taskid`,`file_offset`)
) ENGINE=MyISAM DEFAULT CHARSET=utf8;

--
-- Dumping data for table `node_import_status`
--

INSERT INTO `node_import_status` (`taskid`, `file_offset`, `errors`, `objid`, `status`) VALUES
(1, 22, 'a:0:{}', 365, 2),
(1, 106, 'a:0:{}', 366, 2),
(1, 333, 'a:0:{}', 367, 2),
(1, 474, 'a:0:{}', 368, 2),
(1, 645, 'a:0:{}', 369, 2),
(1, 816, 'a:0:{}', 370, 2),
(1, 951, 'a:0:{}', 371, 2),
(1, 1087, 'a:0:{}', 372, 2),
(1, 1222, 'a:0:{}', 373, 2),
(1, 1307, 'a:0:{}', 374, 2),
(1, 1406, 'a:0:{}', 375, 2),
(1, 1571, 'a:0:{}', 376, 2),
(1, 1688, 'a:0:{}', 377, 2),
(1, 1807, 'a:0:{}', 378, 2),
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(20, 22, 1, 'Editorial Board Members', '<p>\r\n	Patron</p>\r\n<p>\r\n	Prof. Tan Sri Dato&#39; Wira</p>\r\n<p>\r\n	Dr. Sharifah Hapsah Syed Hasan Shahabudin</p>\r\n<p>\r\n	Prof. Dato&#39; Dr. Lokman Saim</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Advisor</p>\r\n<p>\r\n	Prof. Dr. Musalmah Mazlan</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editor-in-Chief Assoc.</p>\r\n<p>\r\n	Prof. Dr. Hanafiah Harunarashid</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editor</p>\r\n<p>\r\n	Prof. Dr. Rohaizak Muhammad</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Associate Editor</p>\r\n<p>\r\n	Assoc. Prof. Dr Srijit Das</p>\r\n<p>\r\n	Assoc. Prof. Dr. Nor Azlin Mohamed Ismail</p>\r\n<p>\r\n	Assoc. Prof. Dr. Razman Jarmin (Surgical Section)</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editorial Board</p>\r\n<p>\r\n	Anatomy</p>\r\n<p>\r\n	Dr. Norzana Abd Ghafar</p>\r\n<p>\r\n	Ophthalmology</p>\r\n<p>\r\n	Dr. Wan Haslina Wan Abd.Halim</p>\r\n<p>\r\n	Radiology</p>\r\n<p>\r\n	Assoc. Prof. Dr. Ahmad Sobri Muda</p>\r\n<p>\r\n	Emergency Medicine</p>\r\n<p>\r\n	Dr. Shahridan Mohd. Fathil</p>\r\n<p>\r\n	Obstetric &amp; Gynaecology</p>\r\n<p>\r\n	Dr. Nirmala @ Chandralega a/p Kampan</p>\r\n<p>\r\n	Orthopaedic</p>\r\n<p>\r\n	Assoc. Prof. Dr. Ya Mohamad Hassan Shukur</p>\r\n<p>\r\n	Pathology</p>\r\n<p>\r\n	Dr. Nurismah Md Isa</p>\r\n<p>\r\n	Anaesthesiology</p>\r\n<p>\r\n	Assoc. Prof. Dr. Choy Yin Choy</p>\r\n<p>\r\n	Surgery</p>\r\n<p>\r\n	Assoc. Prof. Dr. Ismail Sagap</p>\r\n<p>\r\n	Assoc. Prof. Dr. Zamrin Dimon</p>\r\n<p>\r\n	Otorinolaringology</p>\r\n<p>\r\n	Dr. Mazita Ami</p>\r\n<p>\r\n	Local Editors (Advisory Board)</p>\r\n<p>\r\n	Prof. Emeritus Dr. NAfisah Adeeb</p>\r\n<p>\r\n	International Editors (Advisory Board)</p>\r\n<p>\r\n	India</p>\r\n<p>\r\n	Dr.(Ms). Vandana Mehta</p>\r\n<p>\r\n	West India</p>\r\n<p>\r\n	Dr. Vinod Sharma</p>\r\n<p>\r\n	Canada</p>\r\n<p>\r\n	Dr. Joydeep Dutta Chaudhuri</p>\r\n<p>\r\n	United Kingdom</p>\r\n<p>\r\n	Dr. John Murie Dr. Michael G Wyatt</p>\r\n<p>\r\n	Singapore</p>\r\n<p>\r\n	Dr. Chiu Ming Terk</p>\r\n<p>\r\n	Japan</p>\r\n<p>\r\n	Dr. Kazao Shimizu</p>\r\n<p>\r\n	Egypt</p>\r\n<p>\r\n	Dr. Amr Mohsen</p>\r\n<p>\r\n	Australia</p>\r\n<p>\r\n	Dr. B. Sudhir Rao</p>\r\n<p>\r\n	Hong Kong</p>\r\n<p>\r\n	Prof. Dr. David Wong</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Technical Editorial</p>\r\n<p>\r\n	Dr. Badrul Hisham Bahadzor</p>\r\n<p>\r\n	Dr. Zairul Azwan Mohd Azman</p>\r\n<p>\r\n	Nor Hisham Bontak</p>\r\n<p>\r\n	Megat Rafael Maamunor Rashid</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Statistic</p>\r\n<p>\r\n	Assoc. Prof. Dr. Shamsul Azhar Shah</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editorial Assistant</p>\r\n<p>\r\n	Nur Ahlina Abdullah</p>\r\n', '<p>\r\n	Patron</p>\r\n<p>\r\n	Prof. Tan Sri Dato&#39; Wira</p>\r\n<p>\r\n	Dr. Sharifah Hapsah Syed Hasan Shahabudin</p>\r\n<p>\r\n	Prof. Dato&#39; Dr. Lokman Saim</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Advisor</p>\r\n<p>\r\n	Prof. Dr. Musalmah Mazlan</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editor-in-Chief Assoc.</p>\r\n<p>\r\n	Prof. Dr. Hanafiah Harunarashid</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Editor</p>\r\n<p>\r\n	Prof. Dr. Rohaizak Muhammad</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Associate Editor</p>\r\n<p>\r\n	Assoc. Prof. Dr Srijit Das</p>\r\n<p>\r\n	Assoc. Prof. Dr. Nor Azlin Mohamed Ismail</p>\r\n<p>\r\n	Assoc. Prof. Dr. Razman Jarmin (Surgical Section)</p>', '', 1325151107, 1),
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(35, 38, 1, 'M&H new website', '<p>\r\n	M&amp;H is pleased to announce the release of our new website.</p>\r\n', '<p>\r\n	M&amp;H is pleased to announce the release of our new website.</p>\r\n', '', 1341849387, 1),
(36, 39, 1, 'Do you benefit in terms of knowledge by reading the latest issue?', '', '* Yes\n* No\n* Can&#039;t say\n', '', 1339731730, 0),
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(39, 42, 64, 'Editorial Board Members', '<p style="line-height: 13.5pt; margin: 0cm 0cm 0pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Editor-in-Chief</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Srijit Das</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Associate Editors</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Isa Naina Mohamed&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Hatta Sidi</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Section Editors</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	<strong>Basic Sciences</strong></p>\r\n<p style="margin-left:28.35pt;">\r\n	Ahmad Nazrun Shuid</p>\r\n<p style="margin-left:28.35pt;">\r\n	Ruszymah Idrus</p>\r\n<p style="margin-left:28.35pt;">\r\n	Ima Nirwana Soelaiman</p>\r\n<p style="margin-left:28.35pt;">\r\n	Norazlina Mohamad</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<strong>Clinical Sciences</strong></p>\r\n<p style="margin-left:28.35pt;">\r\n	Zaleha Abdullah Mahdy</p>\r\n<p style="margin-left:28.35pt;">\r\n	Ismail Mohd. Saiboon&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Nik Ritza Kosai Nik Mahmood</p>\r\n<p style="margin-left:28.35pt;">\r\n	Tzar Mohd Nizam Khaithir&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Raymond Azman Ali</p>\r\n<p style="margin-left:28.35pt;">\r\n	Raja Affendi Raja Ali</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Editorial Board Members</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Norzana Abdul Ghafar</p>\r\n<p style="margin-left:28.35pt;">\r\n	Chua Kien Hui&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Noraidah Masir</p>\r\n<p style="margin-left:28.35pt;">\r\n	Hamzaini Abdul Hamid</p>\r\n<p style="margin-left:28.35pt;">\r\n	Nik Ruzyanei Nik Jaafar&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Mae-Lynn Catherine Bastion</p>\r\n<p style="margin-left:28.35pt;">\r\n	Fuad Ismail&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Christopher Ho Chee Kong&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Mohd Razif Mohamad Yunus&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Sakthiswary Rajalingham&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>International Advisory Board Members</u></strong></span></p>\r\n<p style="margin-left: 28.35pt;">\r\n	Hassan Mukhtar (USA)</p>\r\n<p style="margin-left: 28.35pt;">\r\n	Colin E. Robertson (UK)</p>\r\n<p style="margin-left: 28.35pt;">\r\n	Rodney Scott (Australia)</p>\r\n<p style="margin-left: 28.35pt;">\r\n	Rajesh Kumar Suri (India)</p>\r\n<p style="margin-left: 28.35pt;">\r\n	Jyoti Arora (India)</p>\r\n<p style="margin-left:28.35pt;">\r\n	Min-Suk Chung (South Korea)</p>\r\n<p style="margin-left:28.35pt;">\r\n	Pasuk Mahakkanukrauh (Thailand)</p>\r\n<p style="margin-left:28.35pt;">\r\n	Bernard Thong Yu Hor (Singapore)</p>\r\n<p style="margin-left:28.35pt;">\r\n	James McLay (UK)</p>\r\n<p style="margin-left:28.35pt;">\r\n	Gregory Tan (Australia)</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Editorial Manager</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Nur Ahlina Abdullah</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<strong><u><span style="color: rgb(0, 0, 255);">Editorial Assistant</span></u></strong></p>\r\n<p style="margin-left:28.35pt;">\r\n	Mohd Firdaus Rosli</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Statistical Consultant</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Azmawati Mohammed Nawi</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Legal Expert</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Nurhusna Zainal Abidin</p>\r\n', '<p style="line-height: 13.5pt; margin: 0cm 0cm 0pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Editor-in-Chief</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Srijit Das</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Associate Editors</u></strong></span></p>\r\n<p style="margin-left:28.35pt;">\r\n	Isa Naina Mohamed&nbsp;</p>\r\n<p style="margin-left:28.35pt;">\r\n	Hatta Sidi</p>\r\n<p style="margin-left:28.35pt;">\r\n	&nbsp;</p>', '', 1465183620, 2),
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(41, 44, 64, 'Guidelines for Authors', '<p>\r\n	<br />\r\n	<em>Medicine and Health</em>accepts articles for publication provided they are original contributions. No articles are published without the signed copyright form.</p>\r\n<div>\r\n	<p>\r\n		<strong>Copyright</strong></p>\r\n	<p>\r\n		Once the article is accepted, the authors have to transfer the rights to the publisher and all matters related to the reproduction of any facts may need prior permission from the publisher. Authors are requested to fill up the Declaration Form before the manuscript is published.</p>\r\n	<p>\r\n		<strong>Conflict of interest and source of funding</strong></p>\r\n	<p>\r\n		The authors should clearly state the conflict of interest (if any) and disclose the source of funding.</p>\r\n	<p>\r\n		<strong>Ethics</strong></p>\r\n</div>\r\n<p>\r\n	Publication in&nbsp;<em>Medicine &amp; Health</em>&nbsp;implies that the authors agree, upon reasonable request, to share any materials or data that are integral to the results presented in the article, including whatever would be necessary for a skilled investigator to verify or replicate the claims. Authors must disclose upon submission any restrictions on the availability of materials or information, such as for patented or dual-purpose materials.</p>\r\n<p>\r\n	Authors must affirm that the research protocol was approved by the appropriate institutional review boards or ethics committees for human (including use of human cells or tissues) or animal experiments and that all human subjects provided appropriate informed consent and/or that regulations concerning the use of animals in research were adhered to.</p>\r\n<p>\r\n	<strong>Plagiarism</strong></p>\r\n<p>\r\n	Plagiarism is a serious offence. All submitted manuscripts are processed through plagiarism checking software. The editor-in-chief reserves the right to take any action once the manuscript is detected for plagiarism.</p>\r\n<div>\r\n	<p>\r\n		<a href="http://medicineandhealthukm.com/sites/medicineandhealthukm.com/files/page/2012/02/Declaration Form.docx" target="_blank">Declaration Form</a></p>\r\n	<p>\r\n		<br />\r\n		Manuscript categories:</p>\r\n</div>\r\n<p>\r\n	1.&nbsp;&nbsp;&nbsp; <strong>Editorials</strong>-These are usually invited or written by experts in their field.</p>\r\n<p>\r\n	2.&nbsp;&nbsp;&nbsp; <strong>Case Report/ Correspondence</strong>- Case report should describe a new case and highlight the differential diagnosis and the challenges encountered while treating. The maximum word count is 2000 words excluding abstract and references.</p>\r\n<p>\r\n	3.&nbsp;&nbsp;&nbsp; <strong>Original Research</strong>- Studies should describe in detail, introduction, methodology, results, discussion, conclusion and acknowledgement (if any). The maximum word count is 6000 words excluding abstract and references.</p>\r\n<p>\r\n	4.&nbsp;&nbsp;&nbsp; <strong>Reviews</strong>- Systematic reviews and meta analysis may be submitted. The authors may liaise with the editor before sending any review article. The maximum word count is 8000 words excluding abstract and references. For systematic reviews and meta-analyses, PRISMA guidelines are to be followed.</p>\r\n<p>\r\n	5.&nbsp;&nbsp;&nbsp; <strong>Letters to the Editor</strong>- Letters pertaining to any published article within 6 weeks of publication, or concerning any important topic may be submitted. Maximum word count is 500 words. The editor-in-chief reserves the right to accept or reject any submission.</p>\r\n<p>\r\n	6.&nbsp;&nbsp;&nbsp; <strong>Clinical Quiz</strong>- Readers and authors are encouraged to send any interesting clinical case for quiz. The case should be described within 750 words and have maximum of 2 figures.</p>\r\n<p>\r\n	7. &nbsp; &nbsp; <strong>Latest Techniques/Innovations/Methods: </strong>Authors who wish to share any latest technique, innovation or methods which may be beneficial for the advancement of medical science, may submit under this section. No particular subheadings are needed but a short abstract within 100 words should be included. The maximum word count should be within 1500 words.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Manuscript format</strong></p>\r\n<p>\r\n	All sections should begin in a new page. The text should be written in Times New Roman, 12 font size.</p>\r\n<p>\r\n	The text should be written in double spaced typing with left and right margins justified. The pages should be numbered at the bottom in the centre.</p>\r\n<p>\r\n	<strong>Title </strong></p>\r\n<p>\r\n	The title should clearly state the type of work done. It is advisable not to use a title which is too long.</p>\r\n<p>\r\n	<strong>Abstract </strong></p>\r\n<p>\r\n	The text should be unstructured and the total word count should not exceed 250 words. An abstract both in English and Bahasa Malaysia (BM) maybe submitted. However, if international authors are unable to submit an abstract in BM, the editorial team may arrange for free services.</p>\r\n<p>\r\n	<strong>Short running title</strong></p>\r\n<p>\r\n	The title page should have a short running title.</p>\r\n<p>\r\n	<strong>Keywords </strong></p>\r\n<p>\r\n	At least 3 keywords should be given as per MeSH style.</p>\r\n<p>\r\n	<strong>Text</strong></p>\r\n<p>\r\n	The remaining sections of the text, which should include Introduction, Materials and Methods, Results, Discussion, and Acknowledgments, and each section should not begin in a new page. Commonly-abbreviated terms should be spelled out in their first occurrence and then may be referenced in abbreviation through the remainder of the manuscript.</p>\r\n<p>\r\n	<em>Introduction.&nbsp;</em>The Introduction section should be written from the standpoint of researchers without specialist knowledge in that area and must clearly state - and, if helpful, illustrate - the background to the research and its aims. Reports of clinical research should, where appropriate, include a summary of a search of the literature to indicate why this study was necessary and what it aimed to contribute to the field. The section should end with a very brief statement of what is being reported in the article.</p>\r\n<p>\r\n	<em>Materials and Methods.&nbsp;</em>Authors should describe experimental and statistical methods in enough detail that other researchers can replicate results and evaluate claims. In general, inclusion of method or reagent details as supplementary material is not acceptable.</p>\r\n<p>\r\n	The sequences of oligonucleotides, if not previously published, should be provided. Novel DNA or protein sequences should be deposited to an appropriate database (eg, Genbank, EMBL, Swiss-Prot), with the accession numbers included in the manuscript. When providing supplier information for material sources, company name and location (city and state, or city and country) should be provided. Website references to company information are not permitted. All novel materials and the procedures to prepare them should be described in sufficient detail to allow their reproduction (eg, DNA constructs, analytical software).</p>\r\n<p>\r\n	<em>Results and Discussion.&nbsp;</em>The Results and Discussion may be combined into a single section or presented separately. Results should present detailed description of the findings in the text and/or tables and figures (but should avoid duplication of findings&#39; description). Results of statistical analysis should include, where appropriate, relative and absolute risks or risk reductions, and confidence intervals. The results and discussion sections may also be broken into subsections with short, informative headings.</p>\r\n<p>\r\n	Discussion, which may usefully start with a brief summary of the major findings, but repetition of parts of the abstract or of the results section should be avoided. Discussion should appraise implications of the findings and place them in the context of prior reports. The focus should be on the importance of these findings to the field and/or patient care.</p>\r\n<p>\r\n	<em>Conclusion.&nbsp;</em>This should state clearly the main conclusions of the research and give a clear explanation of their importance and relevance. Summary illustrations may be included.</p>\r\n<p>\r\n	<em>Acknowledgement.&nbsp;</em>Please acknowledge anyone who contributed towards the study by making substantial contributions to conception, design, acquisition of data, or analysis and interpretation of data, or who was involved in drafting the manuscript or revising it critically for important intellectual content, but who does not meet the criteria for authorship. Please also include their source(s) of funding. Please also acknowledge anyone who contributed materials essential for the study.</p>\r\n<p>\r\n	Authors should obtain permission to acknowledge from all those mentioned in the Acknowledgements.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>References</strong></p>\r\n<p>\r\n	The authors are responsible for the accuracy of the references. References should begin on a new page, be double-spaced and listed according to alphabetical order. Complete author citation is required&nbsp;<strong>(use of &quot;et al&quot; is not acceptable)</strong>. References should conform to the style of the&nbsp;<em>Journal</em>.</p>\r\n<p>\r\n	References in the text to articles, chapters, monographs or other publications should contain the authors&rsquo; names followed by the year of publication without punctuation mark, e.g. (Ismail 1997) or Ismail (1997). References cited only in legends should be numbered in accordance with a sequence established by the first identification in the text of the particular table or illustration.</p>\r\n<p>\r\n	The references themselves should be abbreviated according to the style used in&nbsp;<em>Index Medicus&nbsp;</em>or the&nbsp;<em>World List of Scientific Periodicals</em>, 4th edition. Unpublished observations and personal communications should not be listed but may be inserted in the text. Papers which have been accepted for publication should be included in the list of references as &lsquo;in press&rsquo;. Authors are responsible for verifying the wording of&nbsp;<em>all&nbsp;</em>reference and that references of unpublished work are approved by the original author.</p>\r\n<p>\r\n	<br />\r\n	<em>Medicine &amp; Health&nbsp;</em>reference style:</p>\r\n<table border="1" cellpadding="0" cellspacing="0" style="width:651px;" width="651">\r\n	<tbody>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<strong>In reference list</strong></p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					<strong>In text</strong></p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Article within a journal</em></p>\r\n				<p>\r\n					Koonin, E.V., Altschul, S.F., Bork, P. 1996. BRCA1 protein products: functional motifs.&nbsp;<em>Nat Genet</em>&nbsp;<strong>13:&nbsp;</strong>266-267.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Koonin et al. 1996)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Article within a journal supplement</em></p>\r\n				<p>\r\n					Orengo , C.A. , Bray, J.E., Hubbard, T., LoConte, L., Sillitoe, I. 1999. Analysis and assessment of ab initio three-dimensional prediction, secondary structure, and contacts prediction.&nbsp;<em>Proteins</em>&nbsp;<strong>43</strong>(Suppl 3): 149-170.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Orengo et al. 1999)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>In press article</em></p>\r\n				<p>\r\n					Kharitonov , S.A. , Barnes, P.J. Clinical aspects of exhaled nitric oxide.&nbsp;<em>Eur Respir J</em>, in press.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Kharitonov &amp; Barnes,&nbsp;<em>in press</em>)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Published abstract</em></p>\r\n				<p>\r\n					Zvaifler , N.J., Burger, J.A., Marinova-Mutafchieva, L., Taylor, P., Maini, R.N. 1999. Mesenchymal cells, stromal derived factor-1 and rheumatoid arthritis [abstract].<em>Arthritis Rheum</em>&nbsp;<strong>42:</strong>s250.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Zvaifler et al. 1999)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Article within conference proceedings</em></p>\r\n				<p>\r\n					Jones, X. 1996. Zeolites and synthetic mechanisms. In&nbsp;<em>Proceed-ings of the First National Confe-rence on Porous Sieves: 27-30 June 1996; Baltimore.</em>&nbsp;Edited by Smith Y. Stoneham: Butterworth-Heinemann; 16-27.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Jones 1996)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Book chapter, or article within a book</em></p>\r\n				<p>\r\n					Schnepf, E. 1993. From prey via endosymbiont to plastids: compa-rative studies in dinoflagellates. In&nbsp;<em>Origins of Plastids. Volume 2.</em>&nbsp;2nd edition. Edited by Lewin RA. New York: Chapman and Hall; 53-76.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Schnepf 1993)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Whole issue of journal</em></p>\r\n				<p>\r\n					Ponder, B., Johnston, S., Chodosh, L. (Eds). 1998. Innovative onco-logy. In&nbsp;<em>Breast Cancer Res</em>,&nbsp;<strong>10:</strong>1-72.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Ponder et al. 1998)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Whole conference proceedings</em></p>\r\n				<p>\r\n					Smith, Y. (Ed). 1996.&nbsp;<em>Proceedings of the First National Conference on Porous Sieves: 27-30 June 1996; Baltimore.</em>&nbsp;Stoneham: Butterworth-Heinemann.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Smith 1996)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Complete book</em></p>\r\n				<p>\r\n					Margulis, L. 1970.&nbsp;<em>Origin of Eukaryotic Cells.</em>&nbsp;New Haven: Yale University Press.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Margulis 1970)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Monograph or book in a series</em></p>\r\n				<p>\r\n					Hunninghake, G.W., Gadek, J.E. 1995. The alveolar macrophage. In&nbsp;<em>Cultured Human Cells and Tissues.</em>&nbsp;Edited by Harris, T.J.R. New York: Academic Press; 54-56. [Stoner G (Series Editor):&nbsp;<em>Methods and Perspectives in Cell Biology,</em>&nbsp;vol 1.]</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Hunninghake &amp; Gadek 1995)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Book with institutional author</em></p>\r\n				<p>\r\n					Advisory Committee on Genetic Modification. 1999.&nbsp;<em>Annual Report.</em>&nbsp;London.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Advisory Committee on Genetic Modification 1999)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>PhD thesis</em></p>\r\n				<p>\r\n					Kohavi, R. 1995. Wrappers for performance enhancement and oblivious decision graphs.<em>PhD thesis.</em>&nbsp;Stanford University, Compu-ter Science Department.</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Kohavi 1995)</p>\r\n			</td>\r\n		</tr>\r\n		<tr>\r\n			<td>\r\n				<p>\r\n					<em>Link / URL</em></p>\r\n				<p>\r\n					Hostin, L.O. 1996. Drug use and HIV/AIDS [JAMA HIV/AIDS Web site].</p>\r\n				<p>\r\n					<a href="http://www.amaassn.org/special/hiv">http://www.amaassn.org/special/hiv</a></p>\r\n				<p>\r\n					[26 June 1997].</p>\r\n			</td>\r\n			<td>\r\n				<p>\r\n					(Hostin 1996)</p>\r\n			</td>\r\n		</tr>\r\n	</tbody>\r\n</table>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Tables</strong></p>\r\n<p>\r\n	Tables should be typed double-spaced and submitted on separate files, as part of the manuscript. Tables should be black and white text only and should not include figures or other non-typeset images.</p>\r\n<p>\r\n	Create tables using the table creating and editing feature of your word processing software (e.g. Word, WordPerfect). Do not use Excel or comparable spreadsheet programs. Group all tables in a separate file. Cite tables consecutively in the text, and number them in that order. Each table should appear on a separate page and should include the table title, appropriate column heads, and explanatory legends (including definitions of any abbreviations used). Do not embed tables within the body of the manuscript. They should be self-explanatory and should supplement, rather than duplicate, the material in the text.</p>\r\n<p>\r\n	<strong>Figures</strong></p>\r\n<p>\r\n	Authors should give considerable care in preparing figures. Composites consisting of multiple figures on one page are not allowed. When preparing composite figures, the front side of individual parts should be clearly and properly labelled, must be converted to paths or outlines or they must be embedded in the files. Cite figures consecutively on the site, and number them in the order in which they are discussed.</p>\r\n<p>\r\n	Digital figure (either in JPEG or TIFF file) should be provided for publication purposes. The resolution should be 300 dpi. Figures deviating from these dimensions will be sized at the publisher&#39;s discretion. Unwanted background material should be excluded and edges should be straight.</p>\r\n<p>\r\n	<em>Figure Legends.&nbsp;</em>Legends must be submitted for all figures. They should be brief and specific, and they should appear on a separate manuscript page after the references. Figures may consist of multiple related panels (labeled A, B, C, etc.) described under one figure legend. Use scale markers in the image for electron micrographs, and indicate the type of stain used.</p>\r\n<p>\r\n	<strong>Proof</strong></p>\r\n<p>\r\n	The corresponding author will be contacted by email once proofs are ready. The author should check the proofs carefully, mark any printer&#39;s errors, and answer queries as requested. Author changes should be kept to a minimum. Proof corrections and replacement figures (if any) must be returned within seven days to avoid any delay in publication.</p>\r\n<p>\r\n	<strong>Editorial Communications</strong></p>\r\n<p>\r\n	All correspondence concerning editorial matters should be addressed to the editor-in-chief, at the following address:</p>\r\n<p>\r\n	Medicine &amp; Health,<br />\r\n	Sekretariat Penyelidikan Perubatan &amp; Inovasi,<br />\r\n	Universiti Kebangsaan Malaysia Medical Centre,<br />\r\n	Jalan Yaacob Latif, Bandar Tun Razak<br />\r\n	56000 Cheras, Kuala Lumpur, MALAYSIA,<br />\r\n	Fax:- 603-9146 6634, or<br />\r\n	E-mail:-&nbsp;<a href="mailto:medhealthukm@gmail.com">medhealthukm@gmail.com</a>.</p>\r\n', '<p>\r\n	<br />\r\n	<em>Medicine and Health</em>accepts articles for publication provided they are original contributions. No articles are published without the signed copyright form.</p>\r\n<div>\r\n	<p>\r\n		<strong>Copyright</strong></p>\r\n	<p>\r\n		Once the article is accepted, the authors have to transfer the rights to the publisher and all matters related to the reproduction of any facts may need prior permission from the publisher. Authors are requested to fill up the Declaration Form before the manuscript is published.</p>\r\n	<p>\r\n		<strong>Conflict of interest and source of funding</strong></p>', '', 1458616750, 2),
(111, 114, 1, 'Vol. 4 No. 2 : 2009', '', '', '', 1341217204, 1),
(252, 255, 1, 'Vol. 5 No. 1 : 2010', '', '', '', 1341217138, 1),
(325, 328, 1, 'Submit a Manuscript', '<div class="section">\r\n	<h3>\r\n		<strong>Submit a Manuscript</strong></h3>\r\n	<p>\r\n		<br />\r\n		All articles must be submitted through our submission system.</p>\r\n	<p>\r\n		You need an <strong>author</strong> account to submit a manuscript. If you already have an account (not an author account), please <strong><a href="http://www.medicineandhealthukm.com/user">log in</a></strong> above and apply for the author role from your profile or account page. Or, <strong><a href="http://www.medicineandhealthukm.com/user/register/author">create a new author account</a></strong> to submit a manuscript now. You can then log in to your account to revise or track your submissions.</p>\r\n	<p>\r\n		For any enquiries, do contact us using our online form <strong><a href="http://www.medicineandhealthukm.com/contact">here</a></strong>.</p>\r\n</div>\r\n', '<div class="section">\r\n	<h3>\r\n		<strong>Submit a Manuscript</strong></h3>\r\n	<p>\r\n		<br />\r\n		All articles must be submitted through our submission system.</p>', '', 1346625097, 2),
(327, 330, 1, 'ABOUT', '\r\n', '\r\n', '', 1340897915, 1),
(328, 331, 1, 'Vol. 6 No. 1 : 2011', '', '', '', 1341217072, 1),
(329, 332, 1, 'Vol. 5 No. 2 : 2010', '', '', '', 1341217103, 1),
(330, 333, 64, 'Instruction for reviewers', '', '', '', 1363921512, 1),
(331, 334, 64, 'Article In Press', '\r\n', '\r\n', '', 1381387978, 1),
(332, 335, 1, 'Vol. 1 No. 1 : 2006', '', '', '', 1341217281, 1),
(333, 336, 1, 'Vol. 2 No. 1 : 2007', '', '', '', 1341218182, 1),
(334, 337, 1, 'Vol. 2 No. 2 : 2007', '', '', '', 1341218208, 1),
(335, 338, 1, 'Vol. 3 No. 1 : 2008', '', '', '', 1341218559, 1),
(336, 339, 1, 'Vol. 3 No. 1 suppl : 2008', '', '', '', 1341891511, 1),
(337, 340, 1, 'Vol. 3 No. 2 : 2008', '', '', '', 1341219395, 1),
(338, 341, 1, 'Vol. 6 No. 1 suppl : 2011', '', '', '', 1341891498, 1),
(339, 342, 1, 'Mushawiahti M', '', '', '', 1341228657, 0),
(340, 343, 1, 'Syed Zulkifli SZ', '', '', '', 1341228657, 0),
(341, 344, 1, 'Aida Zairani MZ', '', '', '', 1341228657, 0),
(342, 345, 1, 'Faridah H', '', '', '', 1341228657, 0),
(343, 346, 1, 'Relationship between Central Corneal Thickness and Severity of Open Angle Glaucoma using Optical Coherence Tomography', '<p>\r\n	Central&nbsp; corneal&nbsp; thickness&nbsp; plays&nbsp; a&nbsp; major&nbsp; role&nbsp; in&nbsp; the&nbsp; management&nbsp; of&nbsp; many&nbsp; types&nbsp; of glaucoma. Therefore, our aim is to determine the relationship between the severity of glaucoma&nbsp; measured&nbsp; by&nbsp; optical&nbsp; coherence&nbsp; tomography&nbsp; (OCT)&nbsp; and&nbsp; central&nbsp; corneal thickness (CCT) among normal tension and high tension glaucoma patients. This is an observational&nbsp; cross&nbsp; sectional&nbsp; study&nbsp; on&nbsp; 190&nbsp; patients&nbsp; carried&nbsp; out&nbsp; in&nbsp; Universiti Kebangsaan&nbsp; Malaysia&nbsp; Medical&nbsp; Centre&nbsp; (UKMMC).&nbsp; Three&nbsp; groups&nbsp; of&nbsp; patients&nbsp; were identified;&nbsp; 60&nbsp; normal&nbsp; tension&nbsp; glaucoma&nbsp; (NTG),&nbsp; 61&nbsp; primary&nbsp; open&nbsp; angle&nbsp; glaucoma (POAG)&nbsp; and&nbsp; 69&nbsp; control.&nbsp; Patients&nbsp; were&nbsp; identified&nbsp; based&nbsp; on&nbsp; the&nbsp; glaucomatous&nbsp; visual field&nbsp; changes&nbsp; and&nbsp; previous&nbsp; record&nbsp; of&nbsp; intraocular&nbsp; pressure&nbsp; before&nbsp; treatment.&nbsp; Visual acuity&nbsp; and&nbsp; intraocular&nbsp; pressure&nbsp; measurements&nbsp; were&nbsp; recorded.&nbsp; Specular&nbsp; microscope was used to measure the CCT and the severity of glaucoma was evaluated objectively based&nbsp; on&nbsp; the&nbsp; retinal&nbsp; nerve&nbsp; fibre&nbsp; layer&nbsp; (RNFL)&nbsp; thickness&nbsp; using&nbsp; optical&nbsp; coherence tomography.&nbsp; Results&nbsp; showed&nbsp; NTG&nbsp; patients&nbsp; had&nbsp; significantly&nbsp; thinner&nbsp; cornea, 503.07&plusmn;32.27&micro;m&nbsp; compared&nbsp; to&nbsp; the&nbsp; control&nbsp; group,&nbsp; 517.45&plusmn;31.74&nbsp; &micro;m&nbsp; (p=0.012). However,&nbsp; there&nbsp; was&nbsp; no&nbsp; significant&nbsp; difference&nbsp; between&nbsp; the&nbsp; CCT&nbsp; of&nbsp; POAG&nbsp; and&nbsp; NTG groups (p=0.386).Retinal nerve fibre layer (RNFL) thickness was significantly different between&nbsp; the&nbsp; glaucoma&nbsp; and&nbsp; the&nbsp; control&nbsp; groups&nbsp; (p&lt;0.05&nbsp; in&nbsp; all&nbsp; quadrant).&nbsp; However, correlation between CCT and severity of RNFL thinning was only found for the POAG group&nbsp; but&nbsp; not&nbsp; for&nbsp; the&nbsp; NTG&nbsp; group.&nbsp; (mean&nbsp; RNFL&nbsp; and&nbsp; superior&nbsp; quadrant&nbsp; RNFL; r=0.251,0.401) . This study demonstrated the correlation between CCT and severity of glaucoma in&nbsp; POAG&nbsp; patients but&nbsp; not for the&nbsp; NTG group. Therefore, CCT is related to the severity of POAG-related visual loss.</p>\r\n', '<p>\r', '', 1348714503, 1),
(344, 347, 1, 'Vol. 6 No. 2 : 2011', '', '', '', 1341311869, 1),
(345, 348, 1, 'Tamby MR', '', '', '', 1341295893, 0),
(346, 349, 1, 'Juliana MB', '', '', '', 1341295894, 0),
(347, 350, 1, 'Wan Hamilton WH', '', '', '', 1341295894, 0),
(348, 351, 1, 'Hemlata KG', '', '', '', 1341295894, 0),
(349, 352, 1, 'Masir N', '', '', '', 1341295894, 0),
(350, 353, 1, 'Primary Adenocarcinoma of the Fallopian Tube  Mimicking Pelvic Inflammatory Disease', '<p>\r\n	Primary fallopian tube carcinomas are rare and are difficult to diagnose preoperatively. We&nbsp; describe&nbsp; a&nbsp; fallopian&nbsp; tube&nbsp; carcinoma&nbsp; in&nbsp; a&nbsp; 50-year-old&nbsp; lady&nbsp; who&nbsp; presented&nbsp; with symptoms&nbsp; of&nbsp; pelvic&nbsp; inflammatory&nbsp; disease.&nbsp; Ultrasound&nbsp; examination&nbsp; showed&nbsp; bilateral hydrosalphinx. However,&nbsp; CT scan showed features suggestive of a malignant ovarian tumour.&nbsp; At&nbsp; surgery,&nbsp; a&nbsp; markedly&nbsp; dilated&nbsp; left&nbsp; fallopian&nbsp; tube&nbsp; was&nbsp; found&nbsp; and histopathological&nbsp; examination&nbsp; confirmed&nbsp; the&nbsp; presence&nbsp; of&nbsp; a&nbsp;&nbsp;&nbsp; primary&nbsp; serous adenocarcinoma.&nbsp; The&nbsp; uterus&nbsp; and&nbsp; both&nbsp; ovaries&nbsp; were&nbsp; free&nbsp; of&nbsp; tumour.&nbsp; This&nbsp; report highlights&nbsp; that&nbsp; carcinoma&nbsp; of&nbsp; the&nbsp; fallopian&nbsp; tube&nbsp; should&nbsp; be&nbsp; considered&nbsp; as&nbsp; a&nbsp; differential diagnosis in females who present with lower abdominal pain.</p>\r\n', '<p>\r', '', 1348713509, 1),
(351, 354, 1, 'Hafiza A', '', '', '', 1341297858, 0),
(352, 355, 1, 'Azura AH', '', '', '', 1341297858, 0),
(353, 356, 1, 'Azma RZ', '<p>\r\n	<em>Dr. Raja Zahratul Azma bt. Raja Sabudin, </em><br />\r\n	<em>Department of&nbsp; Pathology, Faculty of Medicine,<br />\r\n	Universiti Kebangsaan Malaysia (UKM),<br />\r\n	Jalan Yaacob Latif, Bandar Tun Razak, 56000<br />\r\n	Kuala Lumpur, Malaysia. </em></p>\r\n', '<p>\r\n	<em>Dr. Raja Zahratul Azma bt. Raja Sabudin, </em><br />\r\n	<em>Department of&nbsp; Pathology, Faculty of Medicine,<br />\r\n	Universiti Kebangsaan Malaysia (UKM),<br />\r\n	Jalan Yaacob Latif, Bandar Tun Razak, 56000<br />\r\n	Kuala Lumpur, Malaysia. </em></p>\r\n', '', 1348884455, 1),
(354, 357, 1, 'Azlin I', '', '', '', 1341297858, 0),
(355, 358, 1, 'Zarina AL', '', '', '', 1341297858, 0),
(356, 359, 1, 'Hamidah NH', '', '', '', 1341297858, 0),
(357, 360, 1, 'Early Lineage Switch from T-Acute Lymphoblastic Leukaemia to Common B-All', '<p>\r\n	Leukaemic&nbsp; stem&nbsp; cells&nbsp; have&nbsp; heterogenous&nbsp; differentiation&nbsp; potential.&nbsp; The immunophenotypes of blast cells are usually consistent throughout the disease course even at relapse.&nbsp; Rarely, blast cells may undergo a &lsquo;lineage switch&rsquo; during the course of disease especially during relapse. We would like to highlight such&nbsp; a&nbsp; case in&nbsp; a 10- year&nbsp; old&nbsp; boy&nbsp; who&nbsp; presented&nbsp; with&nbsp; a&nbsp; two&nbsp; weeks&nbsp; history&nbsp; of&nbsp; lethargy,&nbsp; poor&nbsp; appetite,&nbsp; low grade&nbsp; fever,&nbsp; respiratory&nbsp; distress,&nbsp; cardiac&nbsp; failure,&nbsp; generalized&nbsp; oedema&nbsp; and hepatosplenomegaly.&nbsp; Full&nbsp; blood&nbsp; count&nbsp; showed&nbsp; a&nbsp; leucocyte&nbsp; count&nbsp; of&nbsp; 41.5x109/L&nbsp; and platelet&nbsp; count&nbsp; of&nbsp; 37x109/L.&nbsp; The&nbsp; peripheral&nbsp; blood&nbsp; film&nbsp; showed&nbsp; presence&nbsp; of&nbsp; numerous blast cells. Bone marrow aspiration revealed a hypercellular marrow, which consisted of mainly blast cells with high nuclear to cytoplasmic ratio and inconspicuous nucleoli. Immunophenotyping and cytochemistry results were consistent with the diagnosis of T- cell&nbsp; acute&nbsp; lymphoblastic&nbsp; leukaemia.&nbsp; The&nbsp; patient&nbsp; achieved&nbsp; remission&nbsp; after&nbsp; treatment with&nbsp; UK&nbsp; ALL&nbsp; 97&nbsp; protocol,&nbsp; regime&nbsp; B&nbsp; chemotherapy.&nbsp; However,&nbsp; he&nbsp; relapsed&nbsp; seven months after the initial diagnosis with 26% blast cells in the bone marrow aspirate. The majority was L1 blast cells admixed with some L2 blast cells. Immunophenotyping was consistent&nbsp; with&nbsp; common&nbsp; precursor&nbsp; B&nbsp; acute&nbsp; lymphoblastic&nbsp; leukaemia.&nbsp; The&nbsp; treatment was changed to a more lineage specific chemotherapy. Nonetheless, the patient never achieved remission and was planned for palliative management. This case illustrated a unique and rare case of rapid lineage switch from T-cell acute lymphoblastic leukaemia to common precursor B-cell acute lymphoblastic leukaemia.</p>\r\n', '<p>\r', '', 1344157367, 1),
(358, 361, 1, 'zura', '', '', '', 1355185799, 1),
(359, 362, 58, 'Rahmah MA', '', '', '', 1342672030, 0),
(360, 363, 58, 'Noraishah J', '', '', '', 1342672030, 0),
(364, 367, 1, 'Review of Popliteal  Artery  Entrapment  Syndrome:  A Rare  Cause  of  Claudication  in  A Young Athlete', '', '', '', 1342713490, 0),
(365, 368, 1, 'Breast Self Examination: To Do or Not To Do?', '<p>\r\n	This is a retrospective descriptive study done to look at common presentation and method of detection of breast cancer. A total of 366 case records of patients attending the Breast and Endocrine Clinic at Hospital Kuala Lumpur were reviewed. The peak age of breast cancer presentation was 40 to 49 years (39.6%). Most (81.4%) patients presented with a lump in the breast and the lump was mainly self-detected (97.3%). The mean tumour diameter on presentation was 4.7&plusmn;3 cm. Medical staff detected the disease in 1.6% cases and 1.1% of cases were detected by mammogram. Most women detected the lump themselves, suggesting that Breast Self Examination (BSE) can be used for detection of the disease in places where there is cost and availability constrains for mammogram. Early detection with BSE can possibly offer better treatment options and quality of life despite the evidence that it does not reduce the mortality due to breast cancer.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348739406, 1),
(366, 369, 1, 'Detection of BCR/ABL Gene in Chronic Myeloid Leukaemia: Comparison of Fluorescence in situ Hybridisation (FISH), Conventional Cytogenetics and Polymerase Chain Reaction (PCR) Techniques', '<div>\r\n	<p>\r\n		The reciprocal translocation t(9;22)(q34;q11) which gives rise to the Philadelphia (Ph<sup>1</sup>) chromosome and BCR/ABL fusion gene, plays a pivotal role in the diagnosis and pathogenesis of chronic myeloid leukemia (CML). In this study, we evaluated the role of fluorescence <em>in situ</em> hybridisation (FISH) in detecting the BCR/ABL rearrangement in CML patients. The sensitivity, specificity and detection rate of BCR/ABL gene using FISH, PCR and conventional cytogenetics (karyotyping) methods were also compared. 18 bone marrow samples of patients with clinically diagnosed CML and suspected of CML were collected. The sensitivity, specificity and positive predictive values of FISH were altogether 100% while the sensitivity, specificity and positive predictive values for conventional cytogenetics (karyotyping) were 85%, 100% and 100% respectively. Convetional cytogenetics (karyotyping) detected an additional chromosomal aberration in addition to the Ph<sup>1</sup> chromosome. In conclusion, FISH is a highly sensitive method in detecting the BCR/ABL gene. Conventional cytogenetics (karyotyping) remains an important investigation in the work up of suspected CML patients since there is a possibility of detecting chromosomal aberrations in addition to the Ph<sup>1</sup> translocation. Therefore, conventional cytogenetics (karyotyping) and FISH are complementary techniques and their results should be interpreted together with clinical information.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1348741867, 1),
(367, 370, 1, 'Post-Cardiac Surgery Patient Satisfaction with Quality Nursing Care at Institute Jantung Negara (IJN)', '<p>\r\n	Quality nursing care has a great impact on patient satisfaction. Quality nursing care is perceived by the post cardiac surgery patient, as the degree of physical, emotional and spiritual needs that have to be fulfilled while hospitalisation.&nbsp; The objective of this study was to identify factors in quality nursing care that determine post cardiac patient satisfaction. This single cross-sectional descriptive study using &lsquo;NURSQUAL&rsquo; instrument consisted of four elements; technical competence, information giving, assurance and empathy to measure patient satisfaction with quality nursing care. It consisted of four research questions with 33 items to measure the four elements. Between January to March 2005, 52 post cardiac surgery patients from Institute Jantung Negara (IJN), who fulfilled the inclusion criteria were recruited for this study.&nbsp; There was a significant difference between technical competence, information giving, assurance and empathy with patient satisfaction with p value &lt; 0.05. The element of assurance made the strongest statistically significant unique contribution to the prediction of patient satisfaction with a p value &lt; 0.05.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348808141, 1),
(368, 371, 1, 'Intraoperative Glycaemic Control in Non Insulin Dependent Diabetes Mellitus: A Comparison between Normal Saline and Ringer''s Lactate', '<p>\r\n	Fifty Non Insulin Dependent Diabetes Mellitus (NIDDM) patients undergoing surgery under epidural anaesthesia were studied. All patients received dextrose 5% infusion at 100 ml/hr from the period of fasting until upon arrival to the operation room. Patients were randomly divided into two groups. Patients in Group 1 (n=25) received normal saline while patients in Group 2 (n=25) were given Ringer&rsquo;s lactate. Both groups received their infusion throughout the operative period up to four hours postoperatively. Blood glucose level was measured at baseline, 45 minutes intra operatively and postoperatively at 30 minutes and four hours by using a glucometer. Patients in Group 2 has a larger mean increase in blood glucose level of 1.5 mmol/L between 4 hours postoperatively and baseline compared to 0.96 mmol/L in Group 1. However, this was not statistically significant. There was no difference in the increase of mean glucose level at 30 minutes&nbsp; when compared to baseline. There was a significant increase in mean blood glucose level in both groups in the postoperative period when compared to baseline. This study demonstrated that patients with NIDDM receiving Ringer&rsquo;s lactate has a larger increase in mean blood glucose level compared to those receiving normal saline, but the magnitude is not statistically significant.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348821043, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(369, 372, 1, 'Prevalence of Non-Dengue Thrombocytopaenia among Adult Patients Presenting with Acute Febrile Illness in Primary Outpatient Clinics', '<p>\r\n	A proportion of patients with acute viral fever with thrombocytopaenia does not necessary have&nbsp; dengue&nbsp; infection.&nbsp; Managing&nbsp; them&nbsp; indiscriminately&nbsp; as&nbsp; dengue&nbsp; infection&nbsp; may&nbsp; not&nbsp; be appropriate.&nbsp; The&nbsp; prevalence&nbsp; of&nbsp; this&nbsp; problem&nbsp; is&nbsp; not&nbsp; exactly&nbsp; known.&nbsp; The&nbsp; objective&nbsp; of&nbsp; this study&nbsp; is&nbsp; to&nbsp; determine&nbsp; the&nbsp; prevalence&nbsp; of&nbsp; acute&nbsp; non-dengue&nbsp; febrile&nbsp; thrombocytopaenia among&nbsp; adult&nbsp; patients&nbsp; presenting&nbsp; with&nbsp; acute&nbsp; non-specific&nbsp; febrile&nbsp; illness&nbsp; in&nbsp; an&nbsp; outpatient setting. This was a clinic-based cross sectional study. Consecutive patients presenting with non-specific&nbsp; febrile&nbsp; illness&nbsp; of&nbsp; less&nbsp; than&nbsp; two&nbsp; weeks&nbsp; were&nbsp; selected&nbsp; from&nbsp; the&nbsp; Primary&nbsp; Care Centre of Hospital Universiti Kebangsaan Malaysia (HUKM) and the Batu 9 Cheras Health Clinic. Full blood count was done on the day of visit and dengue serology was done on day five of illness for all patients enrolled. Seventy three patients participated in this study from May&nbsp; to&nbsp; November&nbsp; 2003.&nbsp; Among&nbsp; the&nbsp; patients,&nbsp; 35&nbsp; (47.9%)&nbsp; were&nbsp; noted&nbsp; to&nbsp; have thrombocytopaenia.&nbsp; Fourteen&nbsp; (40%)&nbsp; patients&nbsp; with&nbsp; thrombocytopaenia&nbsp; were&nbsp; serologically negative.&nbsp; The&nbsp; prevalence&nbsp; of&nbsp; non-dengue&nbsp; febrile&nbsp; thrombocytopaenia&nbsp; was&nbsp; 19.2%.&nbsp; A significant&nbsp; number&nbsp; of&nbsp; patients&nbsp; with&nbsp; acute&nbsp; non-specific&nbsp; febrile&nbsp; illness&nbsp; with thrombocytopaenia&nbsp; were&nbsp; negative&nbsp; for&nbsp; dengue&nbsp; serology.&nbsp; These&nbsp; patients&nbsp; should&nbsp; be differentiated from those with acute febrile thrombocytopaenia, as they might differ in their natural history from those with dengue infection, and hence require different management strategies.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348824759, 1),
(370, 373, 1, 'The Importance of Knowing the Potential for Cure among Women Newly Diagnosed with Breast Cancer', '<p>\r\n	A diagnosis of breast cancer among Malaysian women causes many women to seek cure from different health bodies and is a common practice prevalent among Malaysian women. The objective of the study was to explore whether cure is an important information to women newly diagnosed with breast cancer, and to assess if the information on cure were influenced by variables such as ethnic group, age, level of education and social class. A survey design using a structured interview was adopted for this study and nine items of information were presented to 100 women newly diagnosed with breast cancer. Women were asked to choose which information is important and of priority. Thurstone Scaling method was used to produce rank orderings or profiles of information and scale values according to priority. An Analysis of Variance (ANOVA) and t-test was carried out to look at the differences between the mean scores. Information on the likelihood of cure obtained the highest priority. No significant differences were found in terms of educational level, social class, age and ethnic groups in relation to the information on cure. In conclusion, adequate information on cure is important and a priority to all women newly diagnosed with breast cancer, irrespective of ethnic group, age, level of education and social class. The findings of this study will be useful to health care professionals when providing information to women newly diagnosed with breast cancer.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348826147, 1),
(371, 374, 1, 'Polymerase Chain Reaction Analysis as a Rapid Screening Test for Diagnosis of Fragile X Syndrome', '<p>\r\n	Fragile X syndrome is a result of&nbsp; an unstable expansion of (CGG)<sup>n </sup>trinucleotide sequences in the FMR-1 (Fragile X Mental Retardation 1) gene site at Xq27. In a normal person, n ranges from 6 to 40 repeats with an average of 30 repeats, whereas in a mutated FMR1 gene the sequence is repeated several times over (stuttering gene). Full mutation occurs when n equals 200 repeats or more. Where n equals 50 to 200 repeats, it is a premutation. Fragile X occurs when the FMR-1 gene is unable to make normal amounts of usable Fragile X Mental Retardation Protein, or FMRP. The amount of FMRP in the body is one factor that determines the severity of the Fragile X syndrome. A person with nearly normal levels of FMRP usually has mild or no symptoms, while a person with very little or no normal FMRP has more severe symptoms. The mechanism for the role of the FMRP gene is still being researched upon. However, it has been observed that large numbers of repeats (more than 200) inactivates the gene through a process of methylation and when the gene is inactivated, the cell may make little or none of the needed FMRP. Inheritance is X-linked with reduced penetrance and the frequency of occurrence goes up through generations. The phenotypic manifestations of fragile-X syndrome vary and are largely dependent on the size of the mutation or premutation. The identification of the fragile site on G banded metaphases is a time consuming and delicate process requiring experience and skill, however, molecular diagnosis using DNA analysis and Southern blotting, even though expensive, is more specific in determining the presence or absence of the gene. This study was aimed to establish a rapid polymerase chain reaction (PCR) based - touch down PCR, as a screening method for fragile X syndrome. A total of six cases were analysed. Of these, one was a known case of Fragile X (T1) diagnosed by conventional cytogenetics, two were from the latter&rsquo;s family members namely, his mother (T2) and father (T3), and the other two (T4 and T5) were randomly selected from patients presenting with dysmorphic features and delayed development respectively. One normal control (TC) was included. Cytogenetic analyses for detection of the fragile site was carried out in all cases. Two culture systems were used, namely the synchronised lymphocyte culture and the folate - thymidine deficient culture.&nbsp; Stained metaphases from the fragile X cultures were screened for the presence of the fragile site on the X chromosome. G-banded karyotyping was done using an image analyser to exclude presence of chromosomal abnormalities. DNA was extracted from these samples and amplified by touch-down PCR. Cytogenetic analysis revealed a folate-sensitive fragile site in the affected male, but none in the other five samples. G-banded karyotyping exhibited no additional chromosomal abnormalities. All extracted DNA samples were successfully amplified. Five of the samples showed presence of the product at the expected band at 552bp, excluding the presence of an expansion of CGG segment of the FMR-1 gene. The absence of a band in an affected individual, suggested a fully mutated allele of FRAXA (Folate Sensitive Fragile Site at Xq28). We succeeded in establishing a slightly modified touch-down PCR analysis. Our study indicates that PCR testing offers a rapid and specific method for screening of normal allele and full mutation of the fragile X gene.&nbsp; We suggest this technique to be applied as a complementary tool for cytogenetic analysis to detect the FRAXA gene.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348827067, 1),
(372, 375, 1, 'The Role of Conventional and Molecular Cytogenetics in the Diagnosis of Microdeletion Syndromes', '<p>\r\n	In this report we demonstrate the role of fluorescence in situ hybridisation (FISH) and conventional cytogenetic methods in clinically and cytogenetically confirmed cases of microdeletion syndromes. A total of nine cases were referred to the Cytopathology and Cytogenetic Unit, Hospital Universiti Kebangsaan Malaysia (HUKM) from 2002 to 2004. They include three Prader-Willi syndrome, three DiGeorge syndrome, one Williams syndrome, one Miller-Dieker syndrome and one Kallmann syndrome.Blood samples from the patients were cultured and harvested following standard procedures. Twenty metaphases were analysed for each of the cases. FISH analysis was carried out for all the cases using commercial probes (Vysis, USA): SNRPN and D15S10 for Prader-Willi syndrome, LIS1 for Miller Dieker syndrome, ELN for Williams syndrome, KAL for Kallmann syndrome, TUPLE 1 and D22S75 for DiGeorge syndrome. Conventional cytogenetic analysis revealed normal karyotypes in all but one case with structural abnormality involving chromosomes 9 and 22. FISH analysis showed microdeletions in all of the nine cases studied. This study has accomplished two important findings ie. while the FISH method is mandatory in ruling out microdeletion syndromes, conventional cytogenetics acts as a screening tool in revealing other chromosomal abnormalities that may be involved with the disease.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348896866, 1),
(373, 376, 1, 'Biphenotypic Acute Leukemia: A Report of Two Cases', '<p>\r\n	We report two cases of biphenotypic acute leukaemia diagnosed in Hospital Universiti Kebangsaan Malaysia (HUKM), their clinical, haematological characteristics and response to chemotherapy. Both patients are middle-aged ladies who presented with hepatosplenomegaly and high white cell count, mainly composed of blast cells. Their bone marrow aspirations were hypercellular comprising of more than 90% heterogenous blast cells. Cytochemical analyses show &nbsp;more than 3% positivity towards peroxidase, with smaller blasts showing block positivity towards PAS. Immunophenotypically, the blasts showed dual expression of CD 33 and CD 19, CD 19 and CD34, intra CD22, intra TdT and intraMPO. One of the patients showed presence of the Philadelphia chromosome on cytogenetic analysis which was confirmed by Fluorecsence <em>In Situ</em> Hybridisation (FISH). Molecular analysis also showed presence of the BCR-ABL fusion protein. Both patients were given combination chemotherapy consisting of daunorubicin and cytosine arabinoside.However, the patient with positive Philadelphia chromosome BCR-ABL did not achieve morphological remission after induction chemotherapy. In view of the poor prognosis of this disease, both the patients were planned for upfront peripheral blood stem cell transplantation.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348931197, 1),
(374, 377, 1, 'Cochlear Implantation in Children with Labyrinthitis Ossificans.', '<p>\r\n	Evidence of ossification was previously considered a relative contraindication to cochlear implantation.&nbsp; It was considered difficult or impossible to achieve safe electrode insertion because of bony obstruction.&nbsp; Either the electrodes or the inner ear structures could be damaged. Moreover, obstructed scala tympani could limit the number of electrodes that can be inserted. The efficacy of the electrical stimulation was also questioned, as a higher current would be needed on an ossified cochlea. Finally, the neural survival in ossified cochlea is unknown. This may complicate the surgical procedure and affect the long-term outcome. However, depending on the experience of the surgeon, cochlear implantation has been attempted even in grossly ossified cochlea. Here we illustrate that cochlear implantation is safe in labyrinthitis ossificans.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348889106, 1),
(375, 378, 1, 'Detection of Human Papillomavirus, p53 and c-erbB-2 Protein Expression in Juvenile Laryngeal Papillomatosis: A Report of 2 Cases', '<p>\r\n	The association of human papillomavirus (HPV) with juvenile laryngeal papillomatosis has been well documented. We report two cases of juvenile laryngeal papillomatosis and correlated these cases with presence of HPV, p53 and c-erbB-2 proteins. The first case was a one-year-old male patient and the second a six-year-old female patient. Formalin-fixed paraffin-embedded biopsy specimens were tested for the presence of HPV genome by the technique of in situ hybridisation using wide spectrum and type specific biotinylated probes while the immunohistochemical expression of p53 (D07, 1:50) and c-erbB-2 (DAKO A0485, 1:300) proteins were evaluated with commercially available antibodies. Histologically the tumours in both cases showed papillary configuration of squamous papilloma. The first case detected HPV type 6, HPV type 11 and p53 protein expression while the second case showed only HPV type 6. Both cases of HPV showed positive signals confined to the nuclei in the superficial squamous epithelium. The first case showed p53 positivity seen from the basal region up to one third of the epithelium of laryngeal papillomas and the subsequent recent repeat biopsy showed the positivity of p53 had extended throughout the upper layers of the epithelium.&nbsp; Expression of c-erbB-2 protein was not detected in both cases. These findings were similar as in other studies where follow-up of the cases was recommended since they tend to recur.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348909114, 1),
(376, 379, 1, 'Fine Needle Aspiration Cytology of Metastatic Renal Cell Carcinoma - A Case Report', '<p>\r\n	Fine needle aspiration cytology under radiologic guidance for diagnosis of renal cell carcinoma is well established and is increasingly utilized. This is because renal cell carcinoma displays fairly characteristic cellular features permitting correct cytologic identification. We present a case of a 66-year-old man who had advanced renal cell carcinoma with spread to aortic and cervical lymph nodes, lungs and liver. Fine needle aspiration cytology of the para-aortic mass showed tight clusters of malignant cells with abundant and vacuolated cytoplasm consistent with renal cell carcinoma. Histology of the left cervical lymph nodes together with immunohistochemistry findings were consistent with the cytologic diagnosis of metastatic renal cell carcinoma. The patient succumb to his illness three years after the diagnosis was made.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348909174, 1),
(377, 380, 1, 'Reduction of Tear-Drop Poikilocytes in a Case of Myelofibrosis Following Splenectomy', '<p>\r\n	Myelofibrosis is a chronic myeloproliferative disorder characterised by splenomegaly, leukoerythroblastic anaemia, tear-drop poikilocytosis and marrow fibrosis. Splenectomy is indicated for patients requiring frequent transfusions or massive splenomegaly causing distressing symptoms and severe thrombocytopenia secondary to hypersplenism. A 56 year-old lady diagnosed to have primary myelofibrosis in year 2000 was referred to HUKM for further management of her increasing requirement of blood transfusion and <em>massive splenomegaly. She has had two episodes of acute cholecytitis. Investigations done in our hospital showed that her hemoglobin level was 6.4g/dl, white cell count of 23.4x10<sup>9</sup>/l, platelet count 163x10<sup>9</sup>/l and peripheral blood showing leukoerythroblastic picture and tear-drop poikilocytosis. Leukocyte alkaline phosphatase score (NAP) was 184/100 neutrophils. Ultrasound of the abdomen showed massive splenomegaly with multiple gall stones. In view of the frequent transfusion requirements and symptomatic massive splenomegaly, splenectomy and cholecystectomy was performed. A review of her peripheral blood picture, post-splenectomy, showed marked reduction of the tear-drop poikilocytosis and leukoerythroblastosis. We believe that the spleen plays an important role in their formation, but the exact mechanism remains unclear.</em></p>\r\n', '<p>\r', 'Imported with node_import.', 1348908926, 1),
(378, 381, 1, 'Villoglandular Papillary Adenocarcinoma of the Uterine Cervix with Lymph Node Metastasis - A Case Report and Review of the Literature', '<p>\r\n	Villoglandular papillary adenocarcinoma was first described by Young and Scully in 1989 as a distinct entity with the histological features of exophytic growth proliferation, villous and papillary architecture and mild to moderate nuclear atypia. We report one case of villoglandular papillary adenocarcinoma (VGPA) of the uterine cervix with lymph node metastasis and reviewed the clinicopathological features of six other cases reported in the literature. Our patient is the seventh similar reported cases. They ranged in age from 29 to 54 (mean, 41) years. Five had lymphovascular invasion. All except our patient were treated with radical hysterectomy and radiotherapy. She was followed-up for 7 months and unfortunately dieddue to chest infection.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348910485, 1),
(379, 382, 1, 'Two Cases of Retinal Vasculitis in Ocular Tuberculosis Involving Different Parts of the Vascular System', '<p>\r\n	Two case reports of retinal vasculitis in presumptive ocular tuberculosis are presented. Case 1 is a 26-year-old Malay woman who had phlebitis with choroidal lesions and case 2 is a 27 year old Malay woman who had occlusive arteritis. Both subjects had positive Mantoux tests with negative systemic screen. Both responded clinically to anti-tuberculous therapy within days of commencing therapy. There was however concurrent increase in vitritis which decreased following anti-inflammatory doses of oral corticosteroids. These two cases represent a rare mode of presentation of ocular tuberculosis for which a high index of suspicion is needed. Multidrug anti-tuberculous therapy should be combined with oral corticosteroids for effective treatment.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348914116, 1),
(380, 383, 1, 'Hepatitis B Virus Genotypes and Mutations Affecting HBeAg Production: A Malaysian Perspective', '<p>\r\n	Infection by hepatitis B virus (HBV) is a major global health-care problem. HBV is an accepted factor in the elevated risks for liver disease such as cirrhosis and development of hepatocellular carcinoma. This problem is particularly prevalent in the Asia-Pacific region which includes Malaysia. During infection, the hepatitis B e antigen (HBeAg) is produced in the hosts. This antigen is an important serological marker for diagnosing chronic hepatitis B. Seroconversion to anti-body (anti-HBe) corresponds to the improvement of disease prognosis. However, certain mutations such as the core promoter dual mutations (A1762G1764&rarr;T1762A1764), the codon 15 variants (C1858/ T1858) and the precore stop codon mutations (TGG&rarr;TAG) can affect the HBeAg expression. This has diagnostic and clinical implications. Besides that, the HBV can be grouped into eight genotypes (A to H).&nbsp; Moreover, genotypic subtypes and recombinants have been observed as well. Studies have observed that these can differ in their affiliations with the mutations above as well as with disease prognosis.</p>\r\n', '<p>\r', 'Imported with node_import.', 1352111330, 1),
(381, 384, 1, 'Anxiety and Depression among Patients Before and After Percutaneous Coronary Intervention (PCI) at National Heart Institute (NHI)', '<p>\r\n	Percutaneous coronary intervention (PCI) is an invasive diagnostic investigation that may result in high level of anxiety and &ldquo;fear of the unknown&rdquo; among cardiac patients. An increment in anxiety among PCI patients will augment the level of stress and this extreme stress level will then aggravate depression. The objective of this study was to determine the incidence of anxiety and depression before and after PCI. This was a cross-sectional descriptive study. The Hospital Anxiety and Depression Scale (HADS) was used. It measured two elements, namely, the anxiety and depression levels of patients before and after PCI. This study was conducted in the cardiology wards Anggerik and Dahlia, and Day Care of the National Heart Institute, from January to February 2006.&nbsp; A sample of 61 patients before and after PCI who fulfilled the inclusion criteria were recruited in this study. Of these, 40 and 38 patients before and after PCI respectively, had low anxiety levels; while, 47 and 46 patients before and after PCI respectively, had low depression levels. Anxiety and depression levels before and after PCI were not significantly correlated to socio demographic status. However, a significant correlation (p&lt;0.05) was found between anxiety and depression levels before and after PCI. The low levels of anxiety and depression seen among patients before and after PCI at National Heart Institute could possibly due to the adequate facilities and health education program provided by the physician, nurses and cardiac technician.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348947469, 1),
(382, 385, 1, 'A Study On Anxiety And Depression Level Among High Risk Inpatient Pregnant Women In An Obstetric Ward', '<p>\r\n	Pregnancy is perceived by many pregnant mothers as a period of happiness in anticipation of motherhood. Not all pregnant mothers experience cheerfulness as some may experience a high anxiety and depression level for unknown reasons. The purpose of this study was to determine the level of anxiety and depression among high risk pregnant women and the factors that contribute to their level of anxiety and depression. A descriptive cross-sectional study was conducted on 38 high risk mothers whose stay in hospital exceeded more than three days in the obstetric ward of Hospital Universiti Kebangsaan Malaysia. A self assessment questionnaire &ldquo;Hospital Anxiety Depression scale&rdquo; was used to measure the level of anxiety and depression among high risk pregnant women. Of the 38 participants, 16 (42.1%) women experienced a mild level of anxiety and 22 (57.9%) experienced a severe level of anxiety. Seventeen (44.7%) women was classified as having mild depression and 21 (55.3%) severe depression. The factors contributing to the level of anxiety and depression, include those related with &ldquo;<em>lack of information on disease</em>&rdquo;, &ldquo;<em>family matters&rdquo;</em> and &ldquo;<em>finance&rdquo;.</em> High risk pregnant women in this study experienced a significant level of anxiety and depression during their stay in hospital. It is therefore important for nurses and doctors to be aware and sensitive to the influencing factors that cause anxiety and depression as to enable high risk pregnant mothers to enjoy their pregnancy and childbirth.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348976761, 1),
(383, 386, 1, 'Dysmenorrhoea and its Effects on School Activities Among Adolescent Girls in a Rural School in Selangor, Malaysia', '<p>\r\n	The objective of this study is to determine the prevalence of dysmenorrhoea, its associated factors and its effects on school activities among adolescent girls in a secondary school in a rural district of Selangor, Malaysia. This is a cross-sectional study conducted in a public secondary school. A stratified random sampling of 300 female students (12 to 17 years old) from Form one to Form five classes were selected.&nbsp; A self-administered questionnaire consisting of 20-items was used to collect sociodemographic and menstrual data. Pain intensity for dysmenorrhoea was measured by numerical rating scale. The prevalence of dysmenorrhoea was 62.3%. It was significantly higher in the middle adolescence (15 to 17 years old) age group (p=0.003), girls with regular menstrual cycle (p=0.007) and a positive family history (p&lt;0.05). There was no significant association with mean age of menarche and duration of menstruation. The number of school and class absences increased with increasing severity of dysmenorrhoea (p&lt;0.05). The mean pain score was significantly higher in girls who reported to be unable to participate in sports (p=0.008) and with poor concentration in class (p&lt;0.05). Dysmenorrhoea among the adolescent girls was common in this rural school. It had significant negative impact in their school performance and activities.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348977474, 1),
(384, 387, 1, 'Female Sexual Dysfunction Among Malaysian Women in A Primary Care Setting: Does The Frequency of Sexual Activity Matter?', '<p>\r\n	The aim of the study was tocompare sexual functioning among Malaysian women in a primary care setting between those with a low and high frequency sexual intercourse. Across-sectional study on 230 married Malaysian women in a primary-care setting was conducted at the Bandar Tun Razak Clinic, Cheras. A validated Malay version of Female Sexual Function Index questionnaire (MVFSFI) was used to assess the sexual functioning profiles among women with low and high sexual activity. &nbsp;The percentage of women who had sexual intercourse 3-4 times a week, 1-2 times a week and <u>&lt;</u> 1-2 times a month were 13.4%, 44.3 % and 42.4 % respectively. Women with a low frequency of sexual intercourse (Low SI) tended to suffer from more sexual dysfunction, (&chi;&sup2;=28.98, p &lt; 0.001) compared to those with a high frequency of sexual intercourse (High SI) group.Women who were less sexually active (having low frequency intercourse, ie. &le; 1 &ndash; 2 times per week) were found to be less sexually aroused (&chi;&sup2;= 25.9, p&lt; 0.001), less orgasmic (&chi;&sup2;=19.8, p&lt; 0.001), less lubricated during sexual activity (&chi;&sup2;=11.1, p&lt; 0.001),complain of sexual pain (&chi;&sup2;=4.3, p = 0.033) and feels less satisfied sexually (&chi;&sup2;=12.6, p&lt; 0.001).The problem of female sexual dysfunction (FSD) in the Malaysian primary care population with low sexual activity needs to be addressed.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348980605, 1),
(385, 388, 1, 'Fine Needle Aspiration (FNA) Cytology of the Thyroid: A Cyto-Histopathological Study of 361 Cases in Hospital Universiti Kebangsaan Malaysia', '<p>\r\n	Thyroid nodules are common but thyroid malignancies are not. Fine needle aspiration (FNA) cytology is a diagnostic tool used to screen patients with thyroid nodules who require surgery. We study the diagnostic accuracy of FNA as the initial diagnostic modality in the clinical assessment of thyroid nodules. Between January 1995 until December 2000, 2131 FNA of thyroid nodules were performed. Four hundred and forty-one (20.7%) of these were unsatisfactory and 1690 (79.3%) cases were satisfactory for cytological evaluation. Histopathological diagnosis were available for 361 cases. Cyto-histopathological correlation was carried out for these cases. Our results showed a diagnostic accuracy of 96.2% with sensitivity and specificity rates of 87.7% and 98.4% respectively. Our positive predictive value is 93.4% and our negative predictive value is 96.8%.&nbsp; From this study, we conclude that fine needle aspiration is an important initial screening diagnostic tool for the investigation of thyroid nodules.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348987561, 1),
(386, 389, 1, 'Normograms of Ovarian Volume, Uterine Size and Endometrial Thickness in Urban Midlife Malaysia Women', '<p>\r\n	Cancers of the ovary and corpus uteri afflict 5% and 3.6% Malaysian women respectively. Ovarian cancer (OC) remained the deadliest gynaecological malignancy in perimenopausal women mainly due to the lack of symptoms until the disease had spread. Ultrasonography could provide a preliminary screening allowing the clinician to tailor subsequent management and counselling for these women.&nbsp; To support the basis for selective screening, a study on 517 urban disease free women aged 45 years and above, uterus-intact, non-users of HRT was undertaken. This study presented normograms of ovary, uterus and endometrium derived from entry ultrasound assessment. The sample comprised of 58.0% premenopaused and 42.0% postmenopaused women with an average age of 51.27&plusmn;5.35 years old. Over two thirds were Chinese followed by Malays and Indians. The findings indicated that the average uterine size and endometrial thickness (ET) was 7.21&plusmn;1.67x4.36&plusmn;1.30cm and 6.36&plusmn;3.73mm respectively. Premenopausal women had larger uterus compared to those postmenopaused (p&lt;0.0005) and atrophy of the uterus with age and menopause was demonstrable regardless of ethnicity. Likewise, mean ET in the premenopaused was thicker than the postmenopaused (p&lt;0.0005), showing a gradual decrease with age. Early postmenopausal women demonstrated ET of &lt;5mm, eventually reaching 2mm in women &gt;70 years old. From these findings, a cut-off of &lt;5.0mm was considered clinically acceptable in normal postmenopaused regardless of ethnicity. Nearly one fifth (18.9%) had uterine fibroids, a quarter (25%) in premenopausal women as compared to 10% postmenopaused (p&lt;0.0005). The mean right and left ovarian volume (OV) was 5.48&plusmn;7.85cm<sup>3</sup> and 5.80&plusmn;16.78cm<sup>3</sup> respectively. There was a gradual decline in OV with age and menopause from about 7cm<sup>3</sup> at age 45-49 years to less than 1.5cm<sup>3</sup> at 70 years giving a clinical norm OV of &lt;7.5cm<sup>3</sup> and &lt;3.5cm<sup>3</sup> respectively in pre and postmenopausal Malaysian women. Benign ovarian cysts seen in 2.9% had a mean size of 65.14&plusmn;88.22cm<sup>3</sup>. In conclusion, ET and OV normograms should be made available for the Malaysian population to facilitate screening for gynaecological malignancies.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349008700, 1),
(387, 390, 1, 'Prevalence and Reasons for Smoking among Upper Secondary Schoolboys in Hulu Langat, Malaysia', '<p>\r\n	Smoking in adolescence is a great health concern as it is related to many chronic diseases and mortality in later life. It is also associated with other high-risk behaviours among adolescents. The objective of this study was to determine the prevalence and reasons for smoking among upper secondary schoolboys. This was a cross-sectional study conducted among Form 4 and Form 5 students. Three schools in the District of Hulu Langat were identified and students from these schools were selected randomly. The students were given a self-administered questionnaire. A total of 343 students were included in the study. The prevalence of smoking among the schoolboys was 37%. Although the majority of them initiated smoking between 13-15 years old (67%), 21% of them began smoking below 12 years of age (primary school). There was a significant association between adolescent smoking and smoking among family members (p&lt;0.05). The common reasons reported for initiating smoking were curiosity (69%) and peer pressure (51%). However, the common reasons for continuing smoking were stress (70%) and addiction (49%). Schoolwork was reported as the most important factor which contributed to their stress. Smoking prevention programmes should begin early in primary schools. Adolescents should be educated on effective coping strategies in managing stress and learning to be assertive.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349044047, 1),
(388, 391, 1, 'Sharps Injury in Hospital Universiti Kebangsaan Malaysia (HUKM): Experiences of Health Care Workers and Students', '<p>\r\n	Sharps injury among health care workers in HUKM has received serious attention lately due to the possible transmission of diseases like HIV, Hepatits B and Hepatitis C, which can cause serious consequences for the victim. The aim of the study was to gain a deeper understanding of the emotional problems experienced by health care workers who had incurred a sharps injury and the coping strategies adopted by them to overcome the problem. A 28 item semi-structured questionnaire was designed by the supervisors and the researcher as there was no existing questionnaire to be adopted, as no research had been carried out in this area prior to this. The target group consisted of 64 health care workers of different categories working in HUKM. Data was collected over a period of five months from November 2000 to March 2001. The results of the study revealed that the most frequently injured were the staff nurses, nearly 90% of whom were emotionally upset and were worried that they might contract a disease. There was no significant difference in the emotional stress experienced by the different categories of the health care workers, all were equally upset about the injury but each category of staff adopted different methods to overcome the tragedy. Open comments and opinions about prevention and the management of the sharps injury in HUKM as given by the respondents here are taken into account. Several views given by the participants were explored and discussed. Finally, several implications for nursing and recommendations have been presented.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349044952, 1),
(389, 392, 1, 'Work Stress Prevalence among the Management Staff in an International Tobacco Company in Malaysia', '<p>\r\n	A cross sectional study on work stress prevalence was carried out among 185 executives and managers in the head quarter of an international tobacco company. The aim of the study was to identify work stress prevalence in this company and work stressors that were associated with stress experienced by the workers. A questionnaire based on the Personal Stress Inventory by O&#39;Donell (1984) was used. Data collected from participants included sociodemography factors, symptoms of stress and work stressors related to organizational policy, organizational structure, organizational process, and work environment. The study showed that the prevalence of stress among executives was 68.1% and managers were 67.9%. There was no significant difference in the level of stress between the executives and the managers in the company.&nbsp; The sociodemographic factors that were significantly associated with stress were salary, number of children and personal factors. The significant stressors in the workplace were lack of job recognition, over focusing on quality of work, heavy workload and long working hours.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349060608, 1),
(390, 393, 1, 'Kimura’s Disease Mimicking Hodgkin’s Lymphoma: An Unusual Cause of Parotid Swelling', '<p>\r\n	Kimura&rsquo;s disease (KD) is a rare, benign chronic inflammatory disease of unknown aetiology, typically presents in the Orientals as subcutaneous masses in the head and neck region that could be easily misdiagnosed as a malignant tumour, leading to unnecessary radical surgery or intensive cytotoxic therapy. It has been mainly reported in the Chinese and Japanese literature. It is difficult to diagnose before tissue biopsy and fine needle aspiration cytology (FNAC) has limited value. Hence, unless the pathologists are aware of this entity, it might be mistaken as a malignant lesion. We encountered a case of KD in a Malay patient presenting as a parotid mass that was initially diagnosed as Hodgkin&rsquo;s lymphoma (HL). This disorder should be suspected in young male Asian patients presenting with a painless unilateral mass in the head and neck region with associated &nbsp;hypereosinophilia.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349061098, 1),
(391, 394, 1, 'A Comparative Study of Different Induction Techniques (Propofol-Placebo, Propofol-Ephedrine and Propofol-Placebo-Crystalloid) on Intubating Conditions after Rocuronium Administration', '<p>\r\n	This was a prospective randomized double blind controlled study to compare intubating conditions at 60 seconds with rocuronium 0.6 mg/kg by using three different induction techniques: propofol-placebo (PP), propofol-ephedrine (PE) and propofol-placebo-crystalloid (PC). Ninety patients were included and randomly allocated to receive one of the three combinations. The patients were induced using fentanyl 2 &micro;g/kg, followed by propofol 2.5 mg/kg with normal saline as placebo (Group PP and Group PC) or ephedrine 70 &micro;g/kg (Group PE) given over 30 seconds. Subsequently, rocuronium 0.6 mg/kg was given over five seconds and endotracheal intubations were performed 60 seconds later. Intubating conditions were clinically acceptable in all patients except in four patients in PP group, who had poor intubating conditions. The proportion of excellent intubating conditions was significantly highest in Group PE (94%) followed by Group PC (81%) and lowest in Group PP (50%). In conclusion, induction with propofol-ephedrine and propofol-placebo-crystalloid combinations provided significantly better intubating conditions than propofol alone, when rocuronium 0.6 mg/kg was used for intubation at 60 seconds.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349064317, 1),
(392, 395, 1, 'Study of House Officers in a Teaching Hospital on Knowledge, Perception of Skills and Confidence Level in Performing CPR', '<p>\r\n	Cardio-pulmonary Resuscitation (CPR) is important and should be mastered by House Officers (HO). House officers who have just completed their studies are assigned to acute medical and surgical wards. If a patient in the ward has a cardiac arrest (CA), these doctors are usually the first to attend.&nbsp; Therefore an HO must be confident with CPR skills. They must be competent in performing CPR. The authors assessed 26 new HOs from Universiti Kebangsaan Malaysia Hospital (HUKM) with respect to their self-perception about CPR skills, confidence level in performing CPR and knowledge in performing CPR. Knowledge was assessed by a questionnaire. We found that 16 of 26 (61.5%) assessed themselves to have inadequate knowledge and 46.2% had no confidence in performing CPR. The mean score of the written test was 5.7&plusmn; 1.8. Seven out of 26 (27.0%) HOs had incorrect hand placement position for CPR. Only 4 and 9 out of 26 HOs had their sternal paddle and cardiac apex paddle positions correctly placed respectively. In conclusion, knowledge, perception of skills and confidence levels of HOs on CPR are inadequate and need further assessment and improvement. Medical schools need to review their CPR curriculum in order to prepare HOs adequately to work in emergency situations.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349065790, 1),
(393, 396, 1, 'Effectiveness of Home Care Nursing Program (HCNP) on The Activities of Daily Living of Stroke patients', '<p>\r\n	Home Care Nursing Program (HCNP) at HUKM was started in 1998 and<br />\r\n	evaluation of the program was required. The objective of this study is to evaluate the effectiveness of HCNP on stroke rehabilitation at HUKM. A retrospective study using a matched pair design involving 69 stroke patients admitted to HUKM from August 1998 to December 2000 was carried out. The participants of this study were the HCNP group (n=35), and Non- HCNP (n=34). The matching was based on five criteria; age, sex, race, data of admission and severity of disability. The HCNP group was identified through data at the HCNP unit and their medical reports were traced. For the Non-HCNP group, the participants were identified based on criteria which were similar to that of the HCNP group. Based on the criteria identified, there was no significant difference between the two groups. The Activities of Daily Living was measured using the Modified Barthel Index twice; once on admission (based on record) and one post stroke. The t-test was applied to parametric data and non-parametric data; Chi-square was used to detect any significant relationships between the scores of the two groups. Results showed no significant difference (p=0.1990) in ADL on admission and ADL post stroke patients between the two groups. However, there were significant difference (HCNP p=0.000 and Non-HCNP p=0.000) in ADL on admission and post stroke for stroke patients who sought alternative treatment compared to those who had not.</p>\r\n', '<p>\r\n	Home Care Nursing Program (HCNP) at HUKM was started in 1998 and<br />\r', 'Imported with node_import.', 1349066994, 1),
(394, 397, 1, 'Immunohistochemical Study of p53 Expression in Premalignant and Malignant Cervical Neoplasms', '<p>\r\n	One of the most important cervical cancer risk factors is human papillomavirus (HPV) infection. The p53 gene is one of the most important targets of the HPV E6 gene. E6 protein has the ability to stimulate p53 degradation, inhibits several functions of wild-type p53 and it competes with its function including suppression of malignant growth. The aim of this study is to determine the differences in p53 expressions in pre-malignant and malignant cervical neoplasms. This is a retrospective study on 100 cases of cervical neoplasms. There were 21 cases of CIN 1, 8 cases of CIN 2, 25 cases of CIN 3, 36 cases of squamous cell carcinoma, 7 cases of adenocarcinoma and 3 cases of adenosquamous carcinoma. All cases were evaluated by immunohistochemistry using p53 monoclonal antibody.Thirty six of the 54 pre-malignant cases (66.7%) were positive for p53 protein, in contrast to the malignant cases in which, 40 of the 46 cases (87.0%) were positive. The majority of CIN showed absent to focal staining (29/54, 53.7%). In contrast, 84.8% (39/46) of the invasive carcinoma showed regional to diffuse staining.The expression of p53 is greater in the malignant cervical neoplasms than the pre-malignant cervical lesions, suggesting that p53 overexpression is not an early phenomenon in the pathogenesis of cervical cancer. It is also shown to be slightly higher in percentage in CIN 2 and 3 when compared with CIN 1. However, a number of cases were p53 negative, suggesting that other factors may be involved and further HPV studies are indicated.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349067635, 1),
(395, 398, 1, 'Otogenic Brain Abscess: A Retrospective Study of 10 Patients and Review of The Literature', '<p>\r\n	Proper management of chronic otitis media may reduce the incidence of otogenic brain abscess. The aim of this study was to describe the clinical profile, treatment and surgical outcome of patients presenting with otogenic brain abscess. The medical record of patients in Universiti Kebangsaan Malaysia Medical Centre (UKMMC) with otogenic brain abscess were retrospectively analyzed from January 1997-January 2006. Within this period we had approximately 10,800 of follow up cases of chronic otitis media (COM) in our clinic. Ten patients (2 females, 8 males) with an average age of 42 (age range 11 to 69 years) were identified with otogenic brain abscess and included in this study. The mean follow-up period was 14 months.&nbsp; All patients had cholesteatoma. All patients had a history of chronic ear discharge, headache, otalgia and fever. Six of the 10 patients had cerebellar abscess and 4 had temporal lobe abscess.&nbsp; Cerebellar signs were present in 3 patients.&nbsp; All the patients were treated with broad-spectrum antibiotics.&nbsp; In 5 patients, mastoid exploration was the primary surgical treatment and the brain abscesses were treated conservatively.&nbsp; In the other 5 patients, craniotomy and drainage were performed followed by mastoid exploration when their neurological conditions had stabilized. All our patients had uneventful recovery. There were no permanent cerebellar signs during the follow up and no mortality reported in our series. In this series we demonstrated that early diagnosis and proper treatment of otogenic abscess leads to good neurological outcome.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349068650, 1),
(396, 399, 1, 'Placenta Praevia: Preach and Perception', '<div>\r\n	<p>\r\n		Placenta praevia is a known obstetric condition that causes complications to mother and fetus.This study was done to evaluate the knowledge of placenta praevia amongst the obstetric patients. A cross sectional study was carried out in Hospital Ipoh, Perak among 323 antenatal and postnatal patients. Socio-demographic parameters (ie age, race, parity, occupation, educational level) and history of placenta praevia were studied in relation to level of knowledge and attitude towards placenta praevia. Twenty (6.2%) from 323 women had current or past history of placenta praevia. Three had history of placenta praevia while 17 had current placenta praevia with prevalence of 5.3%.The mean score of knowledge<br />\r\n		achieved by patients was 11.8 which indicated overall poor knowledge. Occupation, level of education and history of placenta praevia were found to have a relationship with level of knowledge regarding placenta praevia in all obstetric patients. There was a significant relationship between attitude of patients with current and history of placenta praevia to level of knowledge regarding placenta praevia. (p=0.037, &lt;0.05). In conclusion, the knowledge and attitude towards placenta praevia among obstetric patients in Hospital Ipoh was better in those who had higher education status, white-collar occupation and currently pregnant with placenta praevia.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349070199, 1),
(397, 400, 1, 'Prevalence of Stress and Coping Mechanism among Staff Nurse''s in the Intensive Care Unit', '<div>\r\n	<p>\r\n		The working environment of intensive care unit (ICU) nurses is a constant source of stress. Researchers have described ICU as a stressful environment because of the complex nature of patients&rsquo; health problems requiring an extensive use of very sophisticated technology.&nbsp; This study aimed to identify the prevalence of stress among staff nurses working in ICU, Hospital Universiti Kebangsaan Malaysia (HUKM), factors influencing stress and to explore the symptoms of stress experienced. This descriptive study was conducted on 67 staff nurses working in ICU, HUKM. Data was collected using self-administered questionnaires. The questionnaires included sections on socio-demographic data, factors influencing stress and symptoms of stress experienced. Data was analysed using frequency and percentage. The Chi square test was used to examine the relationshipbetween socio-demographic data and factors influencing stress. Findings indicated that stress symptoms were experienced by 100 per cent (n =67) of staff nurses. Knowledge, working experience, critically ill patients, and environmental factors were one of the many factors contributing to the stress experienced by the ICU nurses in HUKM. Nurses working in ICU, HUKM are found to have a high level of stress. Recommendations like encouraging ICU nurses to take up post basic ICU courses and increasing the nurses quota can reduce the prevalence of stress among ICU nurses. Hospital management has an important role to play in reducing the stress levels of nurses working in ICU as stress has an effect on job performance and quality of nursing care.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349083396, 1),
(398, 401, 1, 'Night Eating Syndrome with Morbid Obesity and Dysthymia: A Case Report - A Psychobiological Approach', '<p>\r\n	The traditional physical and cosmetic-centered model without paying serious attention on the underlying psychosocial issues of care are ill suited to successful treatment outcome of obesity. The objective of this article is to report a case of a retired Malay army sergeant who presented with night eating syndrome (NES) with morbid obesity and dysthymia, and to discuss the psychobiological aspect of the case including to evaluate the effectiveness of the combination treatment of pharmacotherapy and cognitive behavior therapy alongwith diet counseling. The diagnosis was made by using the Structured Clinical Interview Diagnosis (SCID) for DSM-III-R diagnosis and the severity of depression was assessed by Hamilton Depressive Rating Scale. The patient&rsquo;s body mass index was 45, He was found to have dysthymia and the Hamilton Depressive Rating Scale score was 13. We found that the combination of pharmacotherapy, cognitive behavior therapy and nutritional education with the help of the physician proved to be effective in treating morbid obesity with NES and Dysthymia.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349083794, 1),
(399, 402, 1, 'Parapharyngeal Abscess in Immunocompromised Patients', '<p>\r\n	Parapharyngeal abscess is a serious medical condition that may lead to life-threatening complications. Its incidence has dramatically decreased since the advent of antibiotics. We report two cases of parapharyngeal abscesses in immunocompromised patients. We believe that early diagnosis, broad-spectrum antibiotics, surgery and pus drainage can prevent serious complications.</p>\r\n', '<p>\r\n	Parapharyngeal abscess is a serious medical condition that may lead to life-threatening complications. Its incidence has dramatically decreased since the advent of antibiotics. We report two cases of parapharyngeal abscesses in immunocompromised patients. We believe that early diagnosis, broad-spectrum antibiotics, surgery and pus drainage can prevent serious complications.</p>\r\n', 'Imported with node_import.', 1349084925, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(400, 403, 1, 'Pleomorphic Adenoma Originating from Submandibular Salivary Gland in an 8-year-old Girl: A Case Report', '<p>\r\n	Pleomorphic adenoma (PA) typically presents as a benign slow growing, painless neoplasm of the parotid gland. PA arising from the submandibular gland in the paediatric age group is rare. Surgical excision is the treatment of choice. A case of PA in an 8-year-old Malay girl is presented. Clinically she presented with a painless right submandibular mass measuring 2x2cm. A diagnosis of Pleomorphic adenoma was made on Fine Needle Aspiration Cytology (FNAC). She underwent submandibulectomy uneventfully. Postoperatively after two years follow up, she is asymptomatic.</p>\r\n', '<p>\r\n	Pleomorphic adenoma (PA) typically presents as a benign slow growing, painless neoplasm of the parotid gland. PA arising from the submandibular gland in the paediatric age group is rare. Surgical excision is the treatment of choice. A case of PA in an 8-year-old Malay girl is presented. Clinically she presented with a painless right submandibular mass measuring 2x2cm. A diagnosis of Pleomorphic adenoma was made on Fine Needle Aspiration Cytology (FNAC). She underwent submandibulectomy uneventfully. Postoperatively after two years follow up, she is asymptomatic.</p>\r\n', 'Imported with node_import.', 1349085518, 1),
(401, 404, 1, 'Managing Further Rehabilitation in Longer-Term Stroke Patients in the Community: A New Approach.', '<p>\r\n	Stroke is becoming a major public health issue in our country due to the fact that there is an increasing life span of our population. Due to advancement of acute management of stroke, three out of four people will survive beyond the acute phase of stroke. Stroke care providers are still debating regarding the exact period of the terminology &lsquo;longer-term stroke&rsquo;; however many agreed that long-term of stroke refers to the period of one year and thereafter as this period is the determinant for longer-term survival. Management beyond the first year of stroke is complex, encompasses all aspects of patient&rsquo;s life; physical, psychological and integration into community. Rehabilitation being the cornerstone of longer-term stroke management should now focused on more evidence-based approach as to be effective and relevant to the stroke patients.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349096795, 1),
(402, 405, 1, 'A Study of Patient''s Satisfaction with the Emergency Department (ED) of Hospital Universiti Kebangsaan Malaysia (HUKM)', '<p>\r\n	Patients&rsquo; satisfaction is of critical interest to all healthcare providers. Satisfied patients are more likely to seek health care and to comply with prescribed treatment regimes. The objective of the study was to identify factors that influence patient satisfaction with Emergency Department HUKM (ED HUKM). This study was conducted at ED HUKM from January 2007 till March 2007. A convenience sample of 100 participants was recruited from triage 4. The Davis Consumer Emergency Care Satisfaction Scale (CECSS) was adopted and modified. It consists of 19 questions; used a 5 point, Likert type scale of 1 to 5 (1= completely disagree and 5= completely agree) to measure patient satisfaction with triage, health care providers caring behaviours and health teaching. Results showed that 75 participants (75%) were satisfied. There were no significant difference found between male and female patients with total CESCC scores (t=0.308, p values &gt;0.05). Pearson product moment correlation coefficients showed a positive relationship between total and subscale patient satisfaction scores, caring scores (r=0.905, p value &lt;0.05) and teaching scores (r=0.695, p &lt; 0.05). Overall, patients were satisfied with services at the ED HUKM. Patients&rsquo; satisfaction remains as an important quality outcome measure of emergency care in any hospital.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349102010, 1),
(403, 406, 1, 'Coping Styles and Clinical Factors in Relation to Quality of Life among Patients with Schizophrenia', '<div>\r\n	<p>\r\n		This is a cross sectional study examining quality of life in relation to coping styles among<br />\r\n		patients with Schizophrenia (N=92) in remission, from June 2002 to December 2002. Remission state is determined by Brief Psychiatric Rating Scale (BPRS). The psychiatric diagnosis was made by treating psychiatrist using the Clinical Interview Schedule for the DSM-IV Diagnosis. They are subsequently asked to complete demographic and clinical data questionnaire and followed by 36-item short-form health survey (SF-36) of the Medical Outcome Study (MOS) for the assessment of quality of life and the Coping Inventory for Stressful Situation (CISS). The QOL in term of overall mental health among patients with schizophrenia was significantly and positively associated with ethnic group (p&lt;0.05), employment status, type of antipsychotic (p&lt;0.05) and number of admission (p&lt;0.05). Being Chinese and employed are associated with better QOL in term of overall mental health. The commonly used coping style among patients with Schizophrenia is the distraction component of avoidance-oriented coping. The task-oriented coping was significantly and positively correlated with quality of life in term of mental component. There was a negative correlation between the emotion-oriented coping and all the domain of the quality of life. In conclusion, better quality of life is positively correlated with task-oriented coping and inversely related with emotion-oriented coping.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r\n		This is a cross sectional study examining quality of life in relation to coping styles among<br />\r', 'Imported with node_import.', 1349104560, 1),
(404, 407, 1, 'Identification of Y Chromosomal Material in Turner Syndrome by Fluorescence In Situ Hybridisation (FISH)', '<p>\r\n	Turner syndrome is one of the most common chromosomal abnormalities affecting newborn females. More than half of patients with Turner syndrome have a 45X karyotype. The rest of the patients may have structurally abnormal sex chromosomes or are mosaics with normal or abnormal sex chromosomes. Mosaicism with a second X sex chromosome is not usually of clinical significance. However, Turner syndrome patients having a second Y chromosome or Y chromosomal material are at risk of developing gonadoblastoma later in life. The aim of this study is to compare the results of conventional (karyotyping) and molecular cytogenetics (<em>FISH</em>), and discuss the advantages and limitations in the diagnosis of Turner syndrome. We also aim to compare the degree of mosaicism identified using conventional cytogenetics and <em>FISH</em> techniques. Conventional cytogenetics and <em>FISH</em> analyses were performed on eight peripheral blood samples of patients with Turner syndrome collected between 2004 and 2006. From this study, two out of eight patients with Turner syndrome were found to have the <strong>s</strong>ex determining <strong>r</strong>egion on the <strong>Y</strong> chromosome (SRY) gene by <em>FISH</em> analysis. Our results showed that the rate of detection of mosaic cases in Turner syndrome was also increased to 88% after using the FISH technique. We concluded that <em>FISH</em> is more superior to conventional cytogenetics in the detection of the Y chromosomal material. <em>FISH</em> is also a quick and cost effective method in diagnosing Turner syndrome and assessing the degree of mosaicism.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349128472, 1),
(405, 408, 1, 'Knowledge, Attitudes and Sources of Information on Breastfeeding among Pregnant Mothers', '<div>\r\n	<p>\r\n		This is a cross-sectional study on 218 pregnant mothers in an urban government Hospital. The study aimed to assess knowledge, attitudes and sources of information on breastfeeding. The results could be utilised to promote breastfeeding. Almost all the respondents (96.8%) intended to breastfeed their newborns. Most of them (74.8%) were knowledgeable about breastfeeding i.e. colostrum and breast milk was the best food, good for resistance against disease and allergy, filling up stomach easily, helpful in teeth development and maternal recovery after birth, increased bonding, was easy and economical. The two main misconceptions were mothers would stop breastfeeding when infant or mother was sick, and giving clear fluid to the exclusively breastfed infants to prevent dehydration. Most mothers (83.9%) responded positively towards breastfeeding i.e. it was easier than infant formula, had no negative effect on marital relationship or family care, would commence breastfeeding straight after delivery, agreeable to the banning of bottles and teats in hospital and they would not stop breastfeeding even if husband discouraged them. Only 56.9% of the mothers believed they could breastfeed their babies with modesty anywhere. The main sources of information were attained from the mass media (34.9%), antenatal class (32.1%) and other mothers with breastfeeding experiences.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349130308, 1),
(406, 409, 1, 'Prevalence and Risk Factors of Work Related Upper Limb Disorders (WRULD) Among Female Telephone Operators in a Telecommunication Centre in Kuala Lumpur', '<div>\r\n	<p>\r\n		A study was carried out on 79 female telephone operators working in a Telecommuni-cation Centre in Kuala Lumpur to determine the prevalence of Work Related Upper Limb Disorders (WRULD) and its risk factors. Data collection for this study was done between December 2000 and May 2001. The presence of WRULD was determined based on a guided questionnaire and physical examination. The response rate for this study was 94.9% (n= 75) and results showed that the prevalence of WRULD among the female telephone operators was 48.0%: Stage 1= 38.9%, Stage 2= 19.4%, Stage 3= 38.9%, Stage 4= 0% and Stage 5= 2.8%. In the telecommunication centre, it was found that those with WRULD had a mean of 11.59&plusmn;9.09 years of employment duration whereas those without WRULD had a mean of 9.89 &plusmn; 8.48 years of employment duration. None of the factors studied such as Body Mass Index, age, ethnicity, menstrual cycle, hormone replacement therapy, overtime work, duration of employment in the present unit and in the telecommunication centre differ in distribution between subjects with and without WRULD. This could be contributed by factors such as small number of subjects or the common healthy worker effect found in occupational health research.&nbsp; The findings studied of this study show that almost 40.0% of the workers experienced pain and neurological symptoms. Since none of the factors was found to be associated with WRULD, an elaborate study with a case-control design is needed in order to match cases and enable researchers to control the effect of confounding factors. This will ensure risk factors pertinent to WRULD in the Malaysian setting to be identified.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349131667, 1),
(407, 410, 1, 'Recollections Expressed by Mechanically Ventilated Patients of Intensive Care Unit (ICU), Hospital Universiti Kebangsaan Malaysia (HUKM)', '<div>\r\n	<p>\r\n		Ventilated patients who require intensive care unit (ICU) treatment may encounter unpleasant experiences. These experiences may include factual incidents and delusional memories of ICU such as dreams, hallucinations and frightening experiences. A cross sectional study using &ldquo;Intensive Care Experience Questionnaire&rdquo; consisted of four domains: awareness of surrounding, frightening experiences, recall of experience and satisfaction with nursing care. Forty five participants who fulfilled the inclusion criteria were recruited in this study. This study was conducted in ICU of HUKM from January to March 2006. Results showed that 20 respondents (44%) were aware of their surrounding and 31 respondents (69%) reported frightening experiences. Majority of respondents (43 respondents, 96%) reported satisfaction with the delivery of nursing care. There was positive correlation between awareness of surrounding and their abilities to recall their experiences (p&lt;0.05). Patients&rsquo; awareness of surrounding achieved the strongest statistical significance as a contribution to the prediction of their abilities to recall their experiences with beta coefficient value of 0.353 and p&lt;0.05. The finding of this study permits nurses to see the problems of mechanically ventilated patients through reports of unpleasant recollections by the patients themselves. Patients reported frustrations in their attempts to make their needs known. Although they are on sedation, they are aware of all unpleasant events occurring in the ICU</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349139702, 1),
(408, 411, 1, 'UKM Medical Graduates’ Perception of their Communication Skills during Housemanship', '<p>\r\n	The art of talking to patients and their relatives does not come naturally to most of us and the ability to put oneself in the patients&rsquo; predicament is difficult particularly for the young doctors. To identify the communication abilities of the young doctors, a cross sectional study was carried out on 32 house officers who graduated from UKM in 2004 during their house jobs at different hospitals in Malaysia. A standardized questionnaire was used to collect the data. Fifty nine percent respondents claimed that they had communicated very well with patients while 69% with support staff and 88% with peers. On the other hand 38% and 41% of the respondents claimed they communicated very well with their superiors and families of patients. Only 22% of the graduates&rsquo; skills of communication in breaking bad news were very well, while 50% and 81% were very well in counselling patients and taking consent for procedures. Curriculum planners need to emphasize the importance of developing good communication skills in all aspects for the future doctors.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349140301, 1),
(409, 412, 1, 'Utility of Pap Smears and Mammograms in Midlife Urban Malaysian Women', '<p>\r\n	Cancers of the breast and cervix made up 30.4% and 12% of all cancer cases in Malaysia. Thus screening for reproductive organ cancers as women approached menopause becomes exceedingly important. The study reports the baseline assessment tests of 495 disease free urban Malaysian women aged 45 years and above who volunteered in a healthy lifestyle intervention study. The sample comprised of 58.0% premenopaused and 42.0% postmenopaused women with an average age of 51.27&plusmn;5.35 years old. Over two thirds were Chinese followed by Malays and Indians. Overall, abnormal Pap smears<br />\r\n	were seen in 7.6% comprising of 1.3% cervical intraepithelial neoplasia (CIN), 6.1% human papilloma virus (HPV) infection and 0.2% atypical squmous cells of undetermined significances (ASCUS). Yeast and other infections were found in 6.9% and 1.9% respectively. Comparatively, postmenopausal women had a 2.8 fold higher cancerous changes whereas premenopausal women had a higher infection rate, 11.8% vs. 4.7% respectively (p=0.024) with comparable HPV infection rates in both. This study found 1.3% had breast cancer (BC) with 3.6% requiring a biopsy while 3.4% needed regular follow up. Postmenopaused women had more abnormal mammograms (p&lt;0.0005) of a graver nature, requiring monitoring although the rate of BC was lower than the premenopaused, 0.010 versus 0.015. The findings identified better vigorous screening for malignant carcinoma of the cervix in postmenopausal women as the incidence was higher. As for breast abnormalities, screening should begin at an earlier age, amongst pre- and postmenopausal women from 45 years old and above.</p>\r\n', '<p>\r\n	Cancers of the breast and cervix made up 30.4% and 12% of all cancer cases in Malaysia. Thus screening for reproductive organ cancers as women approached menopause becomes exceedingly important. The study reports the baseline assessment tests of 495 disease free urban Malaysian women aged 45 years and above who volunteered in a healthy lifestyle intervention study. The sample comprised of 58.0% premenopaused and 42.0% postmenopaused women with an average age of 51.27&plusmn;5.35 years old. Over two thirds were Chinese followed by Malays and Indians. Overall, abnormal Pap smears<br />\r', 'Imported with node_import.', 1349140892, 1),
(410, 413, 1, 'Anaplastic Large Cell Lymphoma Presenting as a Soft Tissue Mass ', '<p>\r\n	Anaplastic large cell lymphoma (ALCL) is a rare tumour, accounting for approximately 3% of adult non-Hodgkin lymphomas.<sup>1 </sup>Primary systemic ALCL frequently involves both lymph nodes and extranodal sites. A 44-year-old woman presented with a firm, mobile mass in the left iliac fossa region. Ultrasound findings showed a well defined inhomogenous soft tissue mass, measuring 4x4x2.6cm in the deep subcutaneous region. Histopathological examination revealed that the mass was infiltrated by large lymphoid cells with marked nuclear atypia including kidney-shaped nuclei. These neoplastic cells expressed anaplastic lymphoma kinase (ALK) (both nuclear &amp; cytoplasmic staining), CD30 and EMA but not for T-cell (CD45RO and CD3), and B-cell (CD20 &amp; CD79&alpha;) markers. Fluorescence <em>in situ</em> hybridization (FISH) analysis showed a t(2;5)(p23;q35) chromosomal translocation. Subsequently the patient developed shortness of the breath and a thoracic computed tomography (CT) scan showed a mass encasing the right upper lobe bronchus. She also had bilateral axillary lymph nodes, measuring 1 cm in diameter (biopsy was not done). The mediastinum and endobronchial region did not show any abnormalities. She received 6 cycles of CHOP chemotherapy and remained disease free 2 years after diagnosis. ALCL, rarely present as a soft tissue tumour and this disease should be included as a differential diagnosis of any soft tissue mass.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349141658, 1),
(411, 414, 1, 'Clinical Pathway as a Strategy in Improving Healthcare Quality and Cost Containment', '<p>\r\n	The increasing health care cost and public awareness on the delivery of high quality ser&shy;vices has forced healthcare service providers to look into the healthcare delivery system. Clinical Pathway (CP) has been introduced in many hospitals and has been accepted as a beneficial tool in assisting healthcare organizations worldwide. Further, the CP also pro&shy;vides consistently high quality and coordinates services with minimum resources. It is proven to be a beneficial tool in other countries. CP is recently being introduced into the Malaysian healthcare system. The aim of this article was to high&shy;light the benefits of CP in improving healthcare quality and controlling the medical cost. The relevant articles have been reviewed. The majority of literature reviewed con&shy;cluded that there were positive effects in implementing CP. The CP was found to be significant in re&shy;ducing length of stay and medical cost. The introduction of evidence based medicine, clini&shy;cal outcomes, clinical audit, multidisciplinary communication, teamwork and care planning were also improved by CP. The challenges for healthcare providers and healthcare man&shy;agers are to participate and be fully committed in path&shy;way development and implementa&shy;tion in order to improve healthcare quality and cost control.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349148631, 1),
(412, 415, 1, 'Effects of Palm Tocotrienols on Oxidative Stress and Bone Strength in Ovariectomised Rats', '<p>\r\n	Oxidative stress has been associated with postmenopausal osteoporosis which pre&shy;disposes to risk of fracture. Palm tocotrienol is a potent antioxidant and has the poten&shy;tial to be used for treatment of post-menopausal osteoporosis. The aim of the study is to determine if palm tocotrienol supplementation could alleviate oxidative stress in ovariectomised rat model and improve its bone strength. The rats were divided into four groups: (i) sham-operated group (SHAM) (ii) ovariectomised-control group (OVX) (iii) ovariectomised and given 60mg/kg &alpha;-tocopherol by oral gavage (OVX + ATF) (iv) ovariectomised and given 60mg/kg palm tocotrienols by oral gavage (OVX + PTT). After eight weeks of treatment, blood samples were taken to measure oxida&shy;tive status (MDA, SOD and GPX) while the femurs were biomechanically tested for strength and resistance to fracture. Ovariectomy was shown to induce oxidative stress as shown by the raised MDA levels and reduced GPX activity. Palm tocotrienols seemed to offer protection against the ovariectomy-induced oxidative stress as shown by the suppression of MDA levels and raised GPX and SOD activities in the OVX+PTT group. In comparison, &alpha;-tocopherol was only able to raise the SOD but not as high as palm tocotrienols. The biomechanical tests have shown that ovariectomy has not af&shy;fected the bone strength significantly after eight weeks. Palm tocotrienols supplemen&shy;tation for eight weeks was effective in preventing oxidative stress in a post-meno&shy;pausal rat.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349149309, 1),
(413, 416, 1, 'Gamma-tocotrienol Alters Protein Expression of HepG2 Cell Line', '<p>\r\n	Gamma-tocotrienol (GTT) has been shown to exhibit significant antitumor activity in a variety of tumor cells. Previous findings have demonstrated that GTT had antiprolifera&shy;tive effects on a liver cancer cell line (HepG2) with an IC<sub>50 </sub>value of 170&mu;M. In this study, two dimensional gel electrophoresis (2DE) was used to determine changes in protein expression in HepG2 cell line following treatment with GTT. The ultimate aim is to identify the possible molecular mechanisms involved in GTT antitumor activity. This study is focused on obtaining a 2DE protein profile for HepG2 cell line with and without GTT treatment. In the preliminary analysis of the resulting 2DE profiles, 18 protein spots were found to be differentially expressed in cells treated with GTT. This observa&shy;tion is confirmed by extending the analysis to a larger sample size. By studying the effects of GTT treatment on differential protein expression in HepG2 cells the underly&shy;ing mechanisms involved in the antitumor activity of GTT may be elucidated.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349150425, 1),
(414, 417, 1, 'Ginger Extract (Zingiber officinale Roscoe) Triggers Apoptosis in Hepatocarcinogenesis Induced Rats', '<p>\r\n	Ginger extract has been reported previously by our group to exhibit anticancer and an&shy;tioxidant effects by reducing tumour burden and lipid peroxidation respectively in he&shy;patocarcinogenesis induced rats. The current study examined the expression of pro-apoptotic protein caspase-8 and anti-apoptotic protein Bcl-2 in hepatocarcinogenesis treated rats. Thirty normal male Wistar rats were divided into 5 groups based on the diet given: i) control (normal rat chow), ii) olive oil, iii) ginger extract (100mg/kg body weight), iv) choline deficient diet + ethionine, CDE (to induce liver cancer) and v) CDE+ ginger extract. Rats were killed at week 8, and liver tissues were excised for immuno&shy;histochemical study to identify pro-apoptotic and anti-apop&shy;totic proteins, caspase-8 and Bcl-2. The observation on H&amp;E staining confirmed the CDE diet induced liver can&shy;cer as indicated by the presence of numerous oval cells.Identification of Bcl-2 expres&shy;sion showed that 91.6% (11/12) of the samples from the CDE group revealed positive staining while treatment with ginger extract however inhi&shy;bited the expression with only 8.4% (1/12) samples showing positive staining for Bcl-2. As for caspase-8 protein, 41.7% (5/12) of the samples from CDE group showed posi&shy;tive staining, which in&shy;creased to 100% (12/12) with ginger extract treatment. Our findings suggest that gin&shy;ger extract has an anticancer effect by inducing apoptosis in liver cancer cells via up-regulation of the expression of pro-apoptotic protein, caspase-8 and down-regulation of the expression of anti-apoptotic protein Bcl-2.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349151968, 1),
(415, 418, 1, 'Great Teacher Creates Effective Learning Environment: A Study through IIUM Nursing Students'' Eyes', '<p>\r\n	Learning environment in the universities plays an important role in producing highly competent graduates especially in nursing profession. Thus, the most important as&shy;pects are the teaching activities and as well as student &ndash; teacher interaction in daily environment in the university.To investigate the International Islamic University Malay&shy;sia (IIUM) nursing students experience towards their teachers and to identify the rela&shy;tionship between teaching and students learning perception in their learning environ&shy;ment. This study used quantitative method and utilized two out of five subscales in Dundee Ready Educational Environment Measurement (DREEM). The subscales used were students&rsquo; perception of learning (SPoL) , students&rsquo; perception of teacher (SPoT) and total items in these both subscales are 12 and 11 items, respectively. The ques&shy;tionnaire results revealed that IIUM nursing students scored 28.54/48.00 in (SPoL) and 28.13/44.00 in (SPoT). Both findings showed the IIUM nursing students&rsquo; experience their teachers and the learning environment are moving in towards positive directions. The regression finding was 51% of the total variation in students&rsquo; perception of teacher score was explained by students&rsquo; perception of learning. Although the overall sub&shy;scales (SPoL) score in the current study falls in the category of a more positive per&shy;ception, 2 out of 12 items were poorly scored by the IIUM nursing students. The re&shy;searcher strongly agrees that listening to the expression of students is an important consideration for an educational institution. The overall mean score for (SPoT) showed that the students perceived their teachers as moving in the right direction. In this pre&shy;sent IIUM study, one item showed a mean score of less then 2.00<em>.</em> As a result, these two subscales most probably should reflect the same outcome such as in their aca&shy;demic performance and experience greatly during their student life on campus. The arising issues from this DREEM study at IIUM embrace the need for the creation of supportive environment as well as designing and implementing interventions to remedy unsatisfactorily elements of the learning environment for more effective and successful teaching and learning to be realised.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349153281, 1),
(416, 419, 1, 'Information Needs of Post Myocardial Infarction (MI) Patients: Nurse''s Perception in Universiti Kebangsaan Malaysia Medical Centre (UKMMC)', '<p>\r\n	Patient education has been considered as an integral component of care for patient after myocardial infarction. Post myocardial infarction patients require information and knowledge related to their conditions to reduce anxiety and aid recovery. The objective of this study was to identify nurses&rsquo; perception on information needs of post MI patients of UKMMC. A cross sectional study using Cardiac Patient Learning Needs Inventory (CPLNI) adopted from Timmins and Kalizer (2002) was used. It comprises seven cate&shy;gories: related to anatomy and physiology, psychological factor, life style factor, medi&shy;cation information, dietary information, physical activity, and symptom management. This study was conducted in coronary care unit (CCU), coronary rehabilitation ward (CRW), medical ward 1 and medical ward 2 from January 2007 to March 2007. 56 res&shy;pondents (96%) who fulfilled the inclusion criteria were recruited in this study. Results showed that CCU and CRW respondents ranked symptom management as their first ranking followed by medication, life style factor, anatomy and physiology, dietary in&shy;formation, physical activity and psychological factor. However, respondents in Medical Ward 1 and 2 reported and ranked medication information as their top priorities fol&shy;lowed by symptom management, psychological factors, dietary information, physical activity, anatomy and physiology and life style factor. There were significant differ&shy;ences between ward nurses&rsquo; responses with their perception related to anatomy and physiology, psychology factor, life style factor, physical activity, and symptom man&shy;agement with p value&lt;0.05. This study concluded that the information needs are not always perceived in unison by nursing personnel as the CCU and CRW nurses ranked symptoms management as the cardinal factor whilst Medical Ward 1 and 2 nurses give paramount importance to medication. In order to improve this situation for post MI pa&shy;tient, information need has to be tailored, individualized and prioritized based upon their needs.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349154000, 1),
(417, 420, 1, 'Intravenous Iron-Sucrose Complex for Treatment of Iron Deficiency Anaemia in Pregnancy - Experience in an Obstetric Day Care Clinic in a Tertiary Hospital', '<p>\r\n	A cross-sectional study was undertaken to evaluate if outpatient administration of in&shy;travenous iron sucrose complex (Venofer) was a sensible option in treating iron defi&shy;ciency anaemia during pregnancy and puerperium. A total of 120 patients with iron deficiency anaemia were recruited from the Obstetric Day Care Clinic at the Universiti Kebangsaan Malaysia Medical Centre (UKMMC) over 18 months from March 2003 to August 2004. The main outcome measures were haemoglobin increment, patients&rsquo; compliance, adverse effects and saving from hospitalization fees. The pre-treatment haemoglobin (Hb) level was 8.5<u>+</u>0.85g/dl for the antenatal patient and 7.6<u>+</u>0.80 g/dl in the post-partum group. The mean post-treatment haemoglobin increment at day four&shy;teenth was 3.52<u>+</u>0.75g/dl. One patient developed skin rash while another had low-grade pyrexia. Seven patients experienced mild metallic taste. There were no serious side effects or anaphylactic reactions. Ten patients (8.3%) did not complete their ther&shy;apy - eight delivered before completion of treatment; another two defaulted following delivery. The average number of Venofer used was seven ampoules i.e. 700mg per person, most of them required three sessions to complete the course. Outpatient treatment allows each patient to save hospitalization fees of RM45 per day, which to&shy;talled up to RM135 for a 3-days ward stay. An estimation of RM16,200 hospitalization fees for the 120 patients was avoided during the study period. In conclusion, outpatient treatment of anaemia in pregnancy and post-partum period using Venofer was safe and feasible, with high patient compliant and cost-savings from hospitalization fees.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349154494, 1),
(418, 421, 1, 'MRSA Infection in Vascular Surgical Patients: The HKL Experience', '<p>\r\n	Methicillin-resistant <em>Staphylococcus aureus</em> (MRSA) infection is important among vas&shy;cular surgical patients. Its effect can be devastating resulting in limb amputation and mortality. We performed a retrospective patients record analysis to determine the pat&shy;tern of MRSA infection among vascular surgical patients in Hospital Kuala Lumpur from January 2005 to December 2007. We also attempted to identify the factors asso&shy;ciated with poor clinical outcome after such infection. There were 999 patients who underwent vascular surgeries in HKL within the analysis period. Of these 24 patients (2.4%) were detected to have MRSA surgical site infection. The infection was commoner among cigarette smokers, patients with diabetes melitus and those who had previous vascular surgery. Most infections occurred in the emergency surgery category and manifested as wound breakdown. Fifty-four percent of the infected pa&shy;tients ended with graft removal, amputations or death. MRSA infection complicating vascular surgery resulted in poor clinical outcome. This serious threat requires intensi&shy;fied preventive measures.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349159427, 1),
(419, 422, 1, 'The Acquisition, Analyses and Interpretation of fMRI Data: A Study on Functional Specialisation in Primary Auditory Cortices', '<p>\r\n	In this study, functional magnetic resonance imaging (fMRI) is used to investigate func&shy;tional specialisation in human auditory cortices during listening. A silent fMRI paradigm was used to reduce the scanner sound artefacts on functional images. The subject was instructed to pay attention to the white noise stimulus binaurally given at an inten&shy;sity level of 70 dB higher than the hearing level for normal people. Functional speciali&shy;sation was studied using the Matlab-based Statistical Parametric Mapping (SPM5) software by means of fixed effects (FFX), random effects (RFX) and conjunction analyses. Individual analyses on all subjects indicated asymmetrical bilateral activation of the left and right hemispheres in Brodmann areas (BA) 22, 41 and 42, involving the primary and secondary auditory corti&shy;ces. The percentage of signal change is larger in the BA22, 41 and 42 on the right as compared to the ones on the left (<em>p</em>&gt;0.05). The average number of activated voxels in all the respective Brodmann areas are higher in the right hemisphere than in the left (<em>p</em>&gt;0.05). FFX results showed that the point of maximum intensity was in the right BA41 whereby 599&plusmn;1 activated voxels were ob&shy;served in the right temporal lobe as compared to 485&plusmn;1 in the left temporal lobe. The RFX results were consistent with that of FFX. The analysis of conjunction which fol&shy;lowed, showed that the right BA41 and left BA22 as the common acti&shy;vated areas in all subjects. The results confirmed the specialisation of the right auditory cortices in pro&shy;cessing non verbal stimuli.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349187198, 1),
(420, 423, 1, 'Polypharmacy, An Option Prior To Clozapine?: A Case Report', '<p>\r\n	This is a report of a patient on multiple antipsychotic medications for the treatment of schizophrenia. Often, polypharmacy is not encouraged, however, with the advent of newer atypical antipsychotic agents, this practice may need review. This case will be used to highlight the rare instances when polypharmacy may be useful prior to the commencement of clozapine for the treatment of schizophrenia.</p>\r\n', '<p>\r\n	This is a report of a patient on multiple antipsychotic medications for the treatment of schizophrenia. Often, polypharmacy is not encouraged, however, with the advent of newer atypical antipsychotic agents, this practice may need review. This case will be used to highlight the rare instances when polypharmacy may be useful prior to the commencement of clozapine for the treatment of schizophrenia.</p>\r\n', 'Imported with node_import.', 1349187659, 1),
(421, 424, 1, 'Breast Feeding Practice and Knowledge among Mothers Attending an Urban Malaysian Maternal and Child Health Clinic', '<div>\r\n	<p>\r\n		The advantages of breastfeeding have been well established. The objective of this study was to determine the practice and knowledge of breastfeeding in an urban com&shy;munity and identify the influencing factors. This is a cross-sectional study. All mothers attending the Maternal and Child Health Clinic during the study period, who fulfilled the selection criteria, were included in the study. The mothers were interviewed using a self administered questionnaire. A total of 162 mothers participated in the study. The prevalence of initiating breastfeeding during the first week after childbirth was 93.8%. Breastfeeding was found to be higher among Malay mothers (p value &lt;0.001). Mother&rsquo;s education level, employment status, husband&rsquo;s occupation, family income and attending antenatal classes did not show significant association with breastfeeding practice. Influence from significant family members was found to be an important factor towards encouragement of breastfeeding. Overall, the mother&rsquo;s knowledge on breast&shy;feeding was fairly good. However, more than half (58.6%) did not know that they should continue breastfeeding even if the mother or the baby were sick. More than 20% of the mothers were still ignorant regarding &lsquo;lactational amenorrhoea&rsquo;, breast milk protection against allergy and that nipple soreness is common when breastfeeding. Approximately 15% of the mothers were not aware that breast milk is better than for&shy;mula milk. In conclusion, although the mother&rsquo;s knowledge on breast&shy;feeding was fairly adequate, misconception in certain areas of breastfeeding needs correction.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349188707, 1),
(422, 425, 1, 'Induction of DNA Damage and Cell Death by Beta Amyloid Peptide and Its Modification by Tocotrienol Rich Fraction (TRF)', '<p>\r\n	Alzheimer&rsquo;s disease (AD) is associated with increase neuron cell death and decline in cognitive function. This disease is characterized by plaque formation in the brain. It is believed that accumulation of beta amyloid peptide (A&beta;) is an early sequence in the pathophysiology of AD. However, the mechanism of A&beta; toxicity is unknown but may involve increase oxidative stress. This study was thus undertaken to determine the toxic effect of A&beta; on DNA - an important biomolecule which is oxidized by free radicals, and cell apoptosis and its modulation by tocotrienol rich fraction (TRF). Neuroblastoma SH-SY5Y cell lines were treated either with 10&mu;M A&szlig; peptide; 5mg/ml TRF followed by 10&mu;M A&szlig; peptide or 10&mu;M A&szlig; peptide followed by 5mg/ml TRF. Untreated cells served as control. DNA damage was evaluated by the alkaline comet assay, cell viability by the 3-(4,5-dimetiltiazol-2-il)-5-(3-karboksimetoksifenil)2-(4-sulfofenil)-2H-tetrazolium (MTS) and the propidium iodide &amp; calcein-AM staining to determine the number of viable and apoptotic cells. Results showed that A&szlig; peptide induced a significantly higher DNA damage compared to control (p&lt;0.05) and higher number of cell death. However treatment with TRF resulted in significantly less DNA damage, higher cell survival and decreased number of apoptotic cells as compared to A&szlig; peptide treated cells (p&lt;0.05,). Thus this study showed that A&szlig; peptide causes DNA damage and ultimately cell death via apoptosis probably by inducing oxidative DNA damage. This is further supported by the fact that TRF is able to prevent the DNA damage and apoptosis.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349198902, 1),
(423, 426, 1, 'Knowledge, Attitude and Practice of Nurses in Administering Oral Medication at Medical Ward, Universiti Kebangsaan Malaysia Medical Centre', '<div>\r\n	<p>\r\n		Drug administration is an integral part of nurses role in nursing practice. In relation to their diverse roles, nurses are required to have adequate knowledge and good practice. The purpose of this study isto examine the level of knowledge, attitude and practice of nurses in the medical wards administering oral medication and to assess &nbsp;whether nurse&rsquo;s knowledge, attitude and practice is associated with working experience and professional education (post basic training). A cross-sectional study was conducted on forty (40) nurses working in the Medical wards in Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Questionnaires were used to measure the level of knowledge and attitude of nurses and a direct observation technique with checklist to record the practice of nurses. The mean score for knowledge (13.8), attitude (16.4) and practice (10.7) were all within average distribution. No significant difference was found between knowledge and practice of nurses with working experience and post basic training. However, the influence of working experience on attitude of nurses in serving oral medication was found to be significant (p= 0.045). Nurses in medical ward, Universiti Kebangsaan Malaysia Medical Centre, were found to possess an average level of knowledge and attitude in administering oral medication. In addition, this study suggest that work experience of nurses has no influence on knowledge gain and good practice in administering oral medication.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349205908, 1),
(424, 427, 1, 'Melanogenesis Inhibition by Palm Tocotrienol Rich Fraction in Cellular Ageing', '<p>\r\n	Melanin is the pigment that determines skin color. Melanin synthesis is catalysed by the enzyme tyrosinase and is controlled by <em>TYR, TYRP1 </em>and <em>TYRP2</em> genes. In this study, tocotrienol rich fraction (TRF) was used to inhibit melanin synthesis. TRF contains 75% a-tocotrienol and 25% tocopherol. The objective of this study was to determine the effect of tocotrienol rich fraction (TRF) on melanin synthesis by determining melanin content and expression of genes involved in the regulation of melanin synthesis in skin melanocytes. Melanin synthesis was studied by deter&shy;mining melanin level and tyrosinase enzyme activity, while expression of <em>TYR, TYRP1 </em>and <em>TYRP2</em> genes was determined by quantitative real time reverse transcrip&shy;tase poly&shy;merase chain reaction (real time RT-PCR). Primary culture of skin melano&shy;cytes were divided into two groups; control and cells that were treated with 500 &micro;g/ml tocotrienol rich fraction for 24 hours. Our results showed that there was a reduction in melanin content and tyrosinase activity in skin melanocytes treated with tocotrienol rich fraction compared to the control (p&lt;0.05). Expression of <em>TYRP2 </em>gene in melanocytes treated with tocotrienol rich fraction was also decreased (p&lt;0.05) compared to the control. In conclusion, palm tocotrienol rich fraction has anti pigmentation property that inhibits melanin synthesis in cellular aging by inhibiting tyrosinase activity and down regulating <em>TYRP2</em> gene expression.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349215363, 1),
(425, 428, 1, 'Patient Satisfaction on Waiting Time and Duration of Consultation at Orthopedic Clinic, Universiti Kebangsaan Malaysia Medical Centre', '<p>\r\n	Patients are increasingly dissatisfied with the &ldquo;waiting time&rdquo; and &ldquo;duration of consultation&rdquo; at outpatient clinics. A cross sectional study was carried out to measure waiting time and duration of consultation and to examine patients&rsquo; level of satisfaction towards waiting time and duration of consultation at the Orthopedic Clinic, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). This study also examined patient&rsquo;s satisfaction towards the overall clinic services. Data was collected using a self-administered questionnaire. A total of 81 patients participated in this study with a response rate of 100 percent. Findings indicated a significant difference in the mean between expected waiting time level II (from the time the patient is given the calling number till the time patient is called into the doctor&rsquo;s room) and the actual waiting time level II experienced. A significant difference in the mean between the expected duration of consultation and the actual duration of consultation experienced was also noted. However, no significant difference was found between the mean of expected waiting time level I (the time patient register at the counter till the patient is given the calling number) and the actual waiting time (p&gt;0.05). Overall, patient satisfaction towards waiting time was low (29.6%) and duration of consultation was of moderate level (41.9%). The overall satisfaction towards the clinic services were of average level (56.8%). Findings also indicated that patient satisfaction was not influenced by socio-demographic factors of respondents such as age, ethnic, education level, occupation and income level. Important findings from this study may help staff managing the clinic in addressing patients&rsquo; complaint on waiting time and improve patients&rsquo; satisfaction on the overall services of the Orthopedic clinic in UKMMC.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349219516, 1),
(426, 429, 1, 'Survey of Nurses’ Knowledge and Attitude Regarding Post Operative Pain Management at a Teaching Hospital in Malaysia', '<p>\r\n	Post operative pain is an expected adverse outcome following surgery and it often delays mobilization and overall recovery. Acute post operative pain is subjective and cannot be measured directly. The objective of this study was to determine nurses&rsquo; knowledge and attitude towards post operative pain management. A cross sectional study was conducted in surgical wards and the Intensive Care Unit of Universiti Kebangsaan Malaysia Medical Centre from February to April 2008. A 28 item questionnaire which comprised of two domains: knowledge and attitude towards post operative pain management was used. Eighty four respondents successfully responded to the study. Twenty respondents (25%) possessed high level, 58 respondents (69%) possessed moderate level and 5 respondents (6%) had low knowledge level of post operative pain management. Positive attitude towards post operative pain management was reported by 66 respondents (78.5%). There was a significant relationship between nurses&rsquo; academic qualifications and attitude towards post operative pain management (&chi;&sup2; =29.96, p &lt;0.05). In conclusion, nurses in UKMMC possessed moderate level of knowledge and positive attitude towards post operative management. Results of this study show the need to enforce continuing medical education in pain management amongst UKMMC nurses in enhancing the quality of post operative care.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349220070, 1),
(427, 430, 1, 'Waiting Times for Elective Orthopaedic Surgeries in a Teaching Hospital and Their Influencing Factors', '<p>\r\n	Long waiting time for elective operations is a usable key performance index of ineffi&shy;ciency in services. Across-sectional study for orthopedic elective surgery waiting times and their influencing factors was done in a teaching hospital, from January 2003 to June 2004; using case-mix patient&rsquo;s coded records. Main outcomes measured were length of waiting time from first seen at the specialist clinic until surgery (T1) and length of waiting time from the time date of surgery was given until surgery (T2). The median waiting time for T1 was 23 weeks (5.75 months) and 5.86 weeks (1.47 months) for T2. Chi square test was significant for T1 which includes age, comorbidity but only ethnicity was significant for T2. However, multiple logistic regression showed patients paid by third party (OR) of 1.97 (95%CI:1.05-3.72) were significantly likely to wait longer than those who paid out of their own pocket. For T2, women (OR) of 2.29 (95%CI: 1.19-4.42), Indian (OR) of 2.50 (95%CI: 1.16-5.38) and who are self employed (OR) of 4.28 (95%CI: 1.23-14.97) were significantly more likely to have shorter waiting time. Thus predictors for overall waiting time are age, comorbidity, ethnicity, occupa&shy;tion and the method of paying for services rendered by the patients.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349220747, 1),
(428, 431, 1, 'Staphylococcus epidermidis Necrotizing Pneumonia and Pneumatocele in a Term Infant', '<p>\r\n	<em>Staphylococcus epidermidis</em>is emerging as an increasingly important pathogen in the immunocompromised host. We report here a case of<em>S. epidermidis </em>necrotizing pneumonia complicated by pneumatoceles in a term male infant who had earlier undergone surgical repair of tracheo-esophageal fistula. He was treated with intravenous teicoplanin and gentamicin for four weeks followed by two weeks of erythromycin. No percutaneous aspiration or pleural drainage was required. He had complete clinical and radiologic recovery when seen at follow-up six months later. Aggressive antimicrobial therapy for this condition without surgical drainage resulted in complete resolution and avoided complications such as pneumothorax and bronchopleural fistula.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349221130, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(429, 432, 1, 'Role of ABCB1 C3435T Variant in Response to Antiepileptic Drugs in Epilepsy: A Review', '<p>\r\n	Over-expression of P-glycoprotein (P-gp), the encoded product of the ATP-binding cassette (ABC), sub-family B, member 1 (ABCB1/MDR1) gene, plays an important role in mediating multidrug resistance to antiepileptic drugs (AEDs) in about 30% of patients with epilepsy. Genetic variation may in part explain inter-individual differences in phenotype-genotype relationships in the pharmacological response of epilepsy patients to AEDs. The synonymous C3435T polymorphism is one of the most common allelic variants in the ABCB1/MDR1 gene, proposed in the causation of refractory epilepsy. Many studies have shown the relationship between C3435T polymorphism and refractoriness to AEDs in epilepsy. However, there is controversy between the findings of various studies, that is, whether ABCB1/MDR1 C3435T gene polymorphism is associated with response to AEDs in epilepsy patients. This review provides a background and discusses the results of investigations on possible confounding factors affecting the interpretation and implementation of association studies in this area.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349921617, 1),
(430, 433, 1, 'Dental Students'' Knowledge and Perception of Elderly in Relation to Geriatric Dentistry Training', '<p>\r\n	Dental students need to be equipped with skills, knowledge and positive attitudes while providing dental care to the elderly. Reports from Ministry of Health Malaysia indicated that the prevalence of dental caries and periodontal disease was high among older people in the country. The aim of this study was to assess the knowledge and perception of final year dental students towards the elderly in relation to training of geriatric dentistry in Universiti Kebangsaan Malaysia Medical Centre (UKMMC). All the final year dental students were required to complete a questionnaire that comprised of 3 sections: 1) Palmore&rsquo;s Facts on Aging Quiz II, 2) Oral health and social concerns of patients, and 3) Perception towards the teaching of geriatric dentistry. A total of 83 students participated in the study. Twenty-four per cent (n=20) scored more than 70% to questions on knowledge about older people and 73% (n=61) scored between 50-70%. The students had ranked physical disability, transportation problems and complex and overlapping priorities as reasons for elderly not keeping to their appointments. Majority of the students reported that lack of educational resource materials and very minimum clinical sites experience were the main barriers faced during the course. In conclusion, majority of the students had moderate to good knowledge about older people and were able to recognize issues of social concerns that can influence their oral health. In general, there were not many changes in the perceived competencies of managing older patients following the geriatric module.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349924307, 1),
(431, 434, 1, 'Retropharyngeal Abscess: Seven-Year Review - Clinical Presentation, Diagnosis and Management', '<p>\r\n	This was a retrospective study of operative records between 2000 &ndash; 2007 from two ear, nose and throat (ENT) referral centers in Malaysia to review demographic patterns, clinical presentations and management of retropharyngeal abscess (RPA)<strong>. </strong>Our case series comprised of eight patients with five females and three males with a median age at presentation of 47 years. Comorbid factor in this series was diabetes mellitus, with four patients having diabetes. There were three cases of fish bone ingestion. The commonest presentation was dysphagia or odynophagia. None of them had fever. All patients were treated with broad spectrum intravenous antibiotics (IV)&nbsp; and drainage was performed for the abscess. Four patients underwent transoral drainage with three having the cervical approach and one having spontaneous rupture.&nbsp; All patients recovered well from the infection and only one patient passed away due to septicaemia.In conclusion, patients with retropharyngeal abscess may present with vague symptoms of sore throat, odynophagia without fever due to partial antibiotics treatment. A correct diagnosis should be made so that adequate treatment can be given to prevent mortality.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349930955, 1),
(432, 435, 1, 'Riverine Communities and Waterborne Diseases in South Eastern Nigeria', '<div>\r\n	<p>\r\n		Waterborne diseases (WBDs) have continued to be a major health threat to people living in many underdeveloped countries due to absence of basic infrastructure, especially water and health care facilities. In this study we report the findings of an &nbsp;investigation into waterborne diseases amongst the riverine communities in Anambra State, South Eastern Nigeria. A survey of 1200 heads of household using a questionnaire and 600 hospital records were reviewed. A majorconcern of the study was to identify socio-economic and cultural factors that drive the epidemiology of such diseases.Findings revealed significant differences in the prevalence of waterborne diseases within and between the communities, implicating education levels of the head of household, occupation, and source of water used. The study recommends the provision of community health education through trained community health workers and provision of functional and accessible health care services as a primary means of combating water borne diseases.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349931958, 1),
(433, 436, 1, 'Stress and Coping Styles: The Experience of Nursing Staff Working At Two Public Hospitals in Klang Valley, Malaysia', '<div>\r\n	<p>\r\n		Although stress among nursing staff is common, adopting effective coping styles helps in minimizing the problem. The objectives of this study were to compare stress level among nursing staff working in the above disciplines, to identify common coping style used and to determine the relationship between stress and coping styles. This cross-sectional study involved 106 nursing staff who were universally sampled from psychiatric wards and emergency departments in two public hospitals in the Klang Valley. Self-rated questionnaires i.e. Stress Arousal Checklist (SACL) and Coping inventory for Stressful Situations (CISS) were used to assess stress levels and coping styles respectively. There was insignificant difference in terms of stress level between the two nursing staffs. Stress dimension of SACL between the psychiatry (6.53 <u>+</u> 3.18, p=0.372) and emergency (6.02 <u>+</u> 2.67, p=0.372) nursing staffs were insignificant. Arousal dimension of SACL was also insignificant between psychiatry (8.60 <u>+</u> 1.70, p=0.372) and emergency (9.19 <u>+</u> 1.61, p=0.07) nursing staff. Task coping was the most commonly used coping styles among the psychiatry (55.36 <u>+</u> 9.85) and emergency (57.73 <u>+</u> 9.87) nursing staff in this study. Stress dimension of SACL showed weak significant relationship with task coping (r=-0.313, p=0.001) and emotion coping (r=0.292, p=0.001). Arousal dimension of SACL was also found to have weak significant relationship with task coping (r=0.271, p=0.003) and emotion coping (r=-0.251, p=0.005). While nursing was found to be a stressful profession, a significant relationship between stress and coping styles allows intervention to enable better adaptation to the stressful working environment.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349932645, 1),
(434, 437, 1, 'The Role of Powered Instrumentation in the Removal of Antrochoanal Polyps: A 10-Year Review', '<div>\r\n	<p>\r\n		Antrochoanal polyps are rare lesions. We report a series of 40 consecutive cases of antrochoanal polyps treated in our tertiary centre over a period of ten years between May 1998 and April 2008. The median age of the patients was 37 years. The most common clinical symptom was nasal obstruction (92.5%) followed by rhinorrhoea (45%), postnasal drip (35%) and snoring (22.5 %). The median period of follow-up of these patients was 30 months. Various surgical approaches were used; the commonest was powered endoscopic polypectomy and middle meatal antrostomy in 28 patients (70%), followed by endoscopic polypectomy and middle meatal antrostomy in seven patients (17.5%). Five patients (12.5%) underwent powered endoscopic polypectomy without middle meatal antrostomy. In addition, two patients had septoplasty and one had a frontal sinustomy. There were six patients (15%) who had combined sublabial antrostomy. No major complications occurred; four patients relapsed; three patients had initial operation performed elsewhere and one patient developed recurrence after the first surgery. The median hospitalization period was three days. We conclude that, the use of powered instrumentation in the removal of antrochoanal polyps is safe, effective and associated with minimal morbidity.</p>\r\n</div>\r\n', '<div>\r\n	<p>\r', 'Imported with node_import.', 1349945272, 1),
(435, 438, 1, 'Workplace Violence Experienced by Nurses in Universiti Kebangsaan Malaysia Medical Centre', '<p>\r\n	Lately, increasing workplace violence is a problem that requires serious attention by the management of the healthcare sector. This study was carried out to examine workplace violence experienced by nurses in the Universiti Kebangsaan Malaysia Medical Centre. It aimed to identify the category of nursing staff who formed the highest number of victims, the perpetrators and the common types of workplace violence. This was a descriptive cross-sectional study carried out over a three month period on 55 nurses who had participated voluntarily in this study. The demographic data and data on the violent incident were collected by using a 21-item questionnaire. The reliability and validity with Cronbach&rsquo;s alpha was 0.872. Information on the most common types of violence, victims and the perpetrators was also collected. The study revealed that workplace violence among the nursing staff was 3.7% with an average of 1.2% being abused per month and one nurse being abused every other day. The study also revealed that the staff nurses were the most common victims and the perpetrators were mainly the patients (40.6%) and the patients&rsquo; relatives (37.5%). The most common forms of violence were verbal abuse (31.9%) and verbal threat (23.7%). Physical violence was experienced by 44.4% of the victims. These results suggest that workplace violence is a serious problem which should be addressed in order to create a safe environment for the nurses.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349946109, 1),
(436, 439, 1, 'Langerhans cell sarcoma – a rare tumour diagnosed by histomorphology and immunophenotyping', '<div>\r\n	Langerhans cell sarcoma is a rare neoplastic proliferation of Langerhans cells that has overt malignant cytologic features. It behaves more aggressively than its counterpart, Langerhans cell histiocytosis. We present here a case of Langerhans cell sarcoma involving the eighth thoracic vertebra in a 31-yr-old man who presented with a two-month history of back pain. In the past, the diagnosis of Langerhans cell sarcoma relied on the demonstration of Birbeck granules. However, with the advent of monoclonal antibodies suitable on routinely-fixed tissues, the Langerhans cell phenotype can be easily confirmed by CD1a protein expression using immunohistochemistry. In this report, we describe the morphological features and the immunohistochemical findings of this tumour diagnosed even in the absence of Birbeck granules. We also highlighted how immunophenotyping is useful in differentiating Langerhans cell sarcoma from other histiocytic and dendritic tumours.</div>\r\n', '<div>\r', 'Imported with node_import.', 1349947200, 1),
(437, 440, 1, 'Squamous Cell Carcinoma of the Tongue in A Patient with Rothmund-Thomson Syndrome (Recq4 Mutation) - Intolerance to Radiotherapy', '<p>\r\n	We report a case of squamous cell carcinoma of the base of the tongue in a 27 years old patient with Rothmund-Thomson syndrome (RTS). An abnormal DNA repair mechanism in some RTS patients predispose them to malignancy and influence the tolerance to radiotherapy.</p>\r\n', '<p>\r\n	We report a case of squamous cell carcinoma of the base of the tongue in a 27 years old patient with Rothmund-Thomson syndrome (RTS). An abnormal DNA repair mechanism in some RTS patients predispose them to malignancy and influence the tolerance to radiotherapy.</p>\r\n', 'Imported with node_import.', 1349958859, 1),
(438, 441, 1, 'Metaplastic Breast Carcinomas: A Report of Six Cases', '<p>\r\n	Metaplastic breast carcinomas (MBCs) are rare primary breast malignancies characterized histologically by carcinoma of two epithelial types or co-existence of carcinoma with non-epithelial cellular elements. They are aggressive tumours that carry poor prognosis. We reviewed the pathologic features and clinical outcomes of MBCs seen in our institution between the years 2000 to 2007. Out of 471 breast cancer patients, six female patients were histologically-proven to have MBCs giving an incidence rate of 1.3%. The patients comprised four Malays and two Indians and their mean age was 51 years old.&nbsp; Five patients underwent mastectomy (four with axillary clearance and one without) and one had wide local excision with axillary clearance. Axillary lymph node involvement was seen in four patients (three with epithelial only type tumour and one with the biphasic tumour). In all the cases, the tumours were bigger than 5 cm in diameter (T3), grade 3 and estrogen receptor negative. Five patients received chemotherapy while one refused. All of the patients had tumour recurrence with a mean time of recurrence of 9 months. In conclusion, metaplastic breast carcinomas are rare and aggressive tumours usually affect the post menopausal age group. They present as aggressive, large sized, high grade tumours that are estrogen receptor negative. Tumour size and axillary lymph node involvement indicate poor prognosis. Despite treatment with chemotherapy and radiotherapy, these patients have high risk of local recurrence and distant metastases which are potentially fatal.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349958278, 1),
(439, 442, 1, 'Does Chlorella vulgaris modulates the expression of COL and MMP genes in skin ageing?', '<p>\r\n	<em>Chlorella vulgaris</em>, a unicellular microalgae, produces many intracellular phytochemi&shy;cals namely carotenoids, tocopherols, ubiquinone and protein. Skin ageing which is induced by oxidative stress involves decreased extracellular matrix synthesis and in&shy;creased expression of enzymes that degrade the collagenous matrix. The objective of this study was to determine the effect of <em>C. vulgaris</em> on the expression of genes en&shy;coded for collagen (<em>COL</em>) and matrix metalloproteinases (<em>MMPs</em>) which are involved in skin ageing. Human diploid fibroblasts (HDFs) were obtained from circumcision fo&shy;reskin of 8-12 year-old boys. HDFs were cultured into 3 groups: untreated control cells, cells with stress-induced premature senescence (SIPS; cells were induced with H<sub>2</sub>O<sub>2 </sub>at passage 6 for 2 weeks) and SIPS treated with <em>C. vulgaris</em> (prolonged <em>C. vulgaris</em> treatment started at passage 4 and combined treatment with H<sub>2</sub>O<sub>2 </sub>at passage 6 for 2 weeks). Senescence-associated &szlig;-galactosidase (SA &szlig;-gal) was de&shy;termined using senescent cells histochemical staining kit (Sigma, USA). Expression of <em>COLI, COLIII, COLIV, MMPI, MMPII</em> and <em>MMPIII</em> genes was quantitatively analysed with real-time RT-PCR method (iScript&trade; One Step real-time PCR with SYBR&reg; Green; Biorad). HDFs treated with H<sub>2</sub>O<sub>2 </sub>(SIPS) exhibited senescent morphological features of flatten&shy;ing and enlarged with increased expression of SA &szlig;-gal (p&lt;0.05). Gene expres&shy;sion analysis showed <em>COLI</em> was downregulated in SIPS and SIPS treated with <em>C. vulgaris </em>(p&lt;0.05)while <em>COLIII </em>decreased in SIPS and increased in SIPS treated with <em>C. vulga&shy;ris</em> (p&lt;0.05). Expression of <em>MMPI</em> was increased in SIPS and SIPS treated with <em>C. vul&shy;garis</em> (p&lt;0.05) indicating its synergistic effect with H<sub>2</sub>O<sub>2</sub> treatment. In conclusion, in skin ageing,<em>COLI</em> and <em>COLIII</em> genes were downre&shy;gulated while <em>MMPI</em> was upregu&shy;lated. <em>C. vulgaris </em>modulated the expression of <em>COL</em> and <em>MMP </em>genes by downregulat&shy;ing <em>COLI</em> and upregulating <em>COLIII</em> and <em>MMPI</em> but it did not exert anti-ageing effect.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349961383, 1),
(440, 443, 1, 'Four years analysis of Helicobacter pylori infection among patients with dyspeptic at Universiti Kebangsaan Malaysia Medical Centre', '<p>\r\n	<em>Helicobacter pylori</em>has been implicated as an aetiologic agent for type B chronic gastritis, peptic ulcer and gastric cancer. It is considered the most common bacterial infection in the world with approximately 50% of the population being infected. The majority of infected individuals are asymptomatic, with some developing gastritis only. However, chronic infection with <em>H. pylori </em>without antibiotic treatment predisposes infected individuals to the development of gastric cancer. The aim of this study is to determine active <em>H. pylori</em> infection among patients with symptoms of dyspepsia using three combinations of diagnostic methods. In this report, we studied 1,376 consecutive patients who underwent upper gastrointestinal endoscopy at Universiti Kebangsaan Malaysia Medical Center (UKMMC) for dyspepsia from the period January 1999 to December 2002. The classification of patient&rsquo;s diagnosis was assessed by endoscopic and histological examination. The <em>H. pylori</em> status was determined by rapid urease test, histological examination or <em>H. pylori </em>culture. Presence of <em>H. pylori</em> infection was confirmed in 30.8% of patients with dyspepsia. <em>H. pylori</em> infection was more prevalent in older patients and in males compared to females. Patients with severe gastroduodenal diseases were more commonly infected with <em>H. pylori</em>. There was a significant difference in <em>H. pylori</em> prevalence among the different ethnic groups.&nbsp; Indians had the highest infection rate (45.4%), followed by Chinese (36.8%) and the lowest were seen in Malays (18.3%). This finding on determination of active <em>H. pylori</em> infection among patients with dyspepsia is consistent with serological studies that showed racial differences in <em>H. pylori</em> prevalence. However, the pattern of <em>H. pylori</em> infection does not reflect the prevalence of severe gastroduodenal diseases among different ethnic groups.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349962888, 1),
(441, 444, 1, 'Minimal Residual Disease Status in Childhood Acute Lymphoblastic Leukaemias by Flow Cytometry and Their Clinical and Haematological Features', '<p>\r\n	Residual disease in patients with acute leukaemia indicates unfavorable prognosis. The evaluation of remission using flow cytometry allows a better estimation of minimal residual disease (MRD) after induction chemotherapy in childhood acute lymphoblastic leukaemia (ALL) cases. Patients in morphological marrow remission with presence of blast cells of less than 5%, may still have up to 10<sup>10</sup> leukaemic cells. However with flow cytometric analysis, lower levels of the residual leukaemic cells&nbsp; (1 in 10<sup>4</sup> cells) can be detected and it can be used as a tool to predict relapse. This study compared the presenting clinical and haematological features of children with ALL and their residual disease status determined by flow cytometry. Analysis of their MRD status following remission-induction chemotherapy were done at day-28, week-12 and week-20. The cases were also followed up to five years, to determine their survival status. Their residual disease status by flow cytometric immunophenotyping was also compared with their bone marrow findings morphologically. Thirty-eight cases of precursor B-ALL in pediatric patients from UKM Medical Centre (UKMMC) were analyzed. There was no significant correlation between demographic, clinical and haematological features with MRD status at day-28. However, there was a significant correlation between MRD status by flow cytometry and by morphological marrow examination at week-12. Three cases showed persistent MRD findings until week-20 where two of the cases relapsed and died subsequently.&nbsp; Twenty four patients were still alive after five years of follow up.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349963945, 1),
(442, 445, 1, 'Risk Determinants of Peripheral Neuropathy in Patients with Type II Diabetes Mellitus Attending Follow-Up Clinics at University Kebangsaan Malaysia Medical Center (UKMMC): A Cross Sectional Study', '<p>\r\n	Peripheral neuropathy is highly associated with foot complications among diabetics. This study aimedto identify risk factors associated with the development of peripheral neuro&shy;pathy in diabetic patients and their association with degree of severity of peri&shy;pheral neu&shy;ropathy. Across-sectional study was conducted in follow-up clinics at the Uni&shy;versiti Ke&shy;bangsaan Malaysia Medical Centre (UKMMC), Malaysia involving 72 diabetic patients and 19 controls. Exclusion criteria were those with amputated limbs, gross foot deformity and existing peripheral neuropathy. Controls were non diabetics who walked normally, had no history of foot problem and attended the clinic as sub&shy;jects&rsquo; companion. Quantita&shy;tive assessment of neuropathy was done using Semmes-Weinstein monofila&shy;ment. Neu&shy;ropathy Disability Score (NDS) were used to quantify se&shy;verity of diabetic neu&shy;ropathy. &nbsp;Spearman&rsquo;s Rank test and Mann-Whitney test were used to determine correlation be&shy;tween variables and their differences. Logistic regression analysis was used to deter&shy;mine risk factors associated with peripheral neuropathy. The mean HbA1c among di&shy;abetics was 8.6% <u>+</u> 4.1, and mean NDS was 7.0 <u>+</u> 6.0. A total of 79.1% demonstrated various level of neuropathy with presence of callus was asso&shy;ciated with higher NDS scores. Older age (<em>P</em>=0.02), body weight (<em>P</em>=0.03), HbA1c (<em>P</em>=0.005) and duration of di&shy;abetes (<em>P </em>&lt;0.005) showed positive correlation with NDS. Proper foot care program for diabetics should include recognition of the callus, with special emphasis given to those with heavier weight and increasing age.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349967515, 1),
(443, 446, 1, 'Acute mastoid abscess secondary to partially treated upper respiratory tract infection. A case report', '<p>\r\n	Acute mastoid abscess is common in daily practice. In children, it is usually caused by unsuccessful treatment or partially treated acute otitis media (AOM). Some authors had reported that chronic suppurative otitis media (CSOM) can be the source of infection but it is usually associated with the presence of cholesteatoma. A case of an acute mastoid abscess in a 7 year old girl is presented. She had a history of severe otalgia with left post auricular swelling for 3 days. Clinically she was febrile, the left otoscopy showed diffuse post auricular swelling and sagging of the posterior wall of the external canal. She underwent an emergency cortical mastoidectomy for the left mastoid abscess and had an uneventful recovery. In conclusion, this patient was partially treated with antibiotics which increased the risk for &lsquo;masked mastoiditis&rsquo;, and she later developed a mastoid abscess. Therefore we advocate that all AOM patients should be treated with antibiotics at least for a duration of one week.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349967797, 1),
(444, 447, 1, 'Concomitant t(8;21) And Trisomy 4 in a Patient with Acute Myeloid Leukemia (AML)', '<p>\r\n	The t(8;21)(q22;q22) is a frequently occurring aberration in acute myeloid leukemia (AML) (18-20%) and usually correlate with French-America-British (FAB) M2 subtype.&nbsp; Several studies showed that patients carrying this abnormality demonstrated good response to standard chemotherapy but also have a high incidence of disease relapse. Trisomy 4 is a rare and specific chromosomal abnormality occurring in AML M2 or M4 of the FAB subtypes. We report a case of a 33-year-old female with an apparently clinical and hematologic diagnosis of acute promyelocytic leukemia (APL) in whom cytogenetic analysis revealed an abnormal karyotype with trisomy 4, in addition to t(8;21). Trisomy 4 and t(8;21) in a patient with AML is rare. The significance of t(8;21) with trisomy 4 in AML are unclear but patients bearing this abnormality are associated with a poor prognosis.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349968221, 1),
(445, 448, 1, 'Thymoma: A case report and review of the literature', '<p>\r\n	Thymomas comprise about 1% of all mediastinal tumours and are rare in children. Typically, these tumours are aggressive, with a poor outcome. The current treatment of invasive thymoma is often multidisciplinary. We report a 16-year-old boy with invasive thymoma who was successfully treated with systemic chemotherapy, surgical resection and irradiation. The patient has been in continuous remission for 6 years without radiographic evidence of tumour recurrence.</p>\r\n', '<p>\r\n	Thymomas comprise about 1% of all mediastinal tumours and are rare in children. Typically, these tumours are aggressive, with a poor outcome. The current treatment of invasive thymoma is often multidisciplinary. We report a 16-year-old boy with invasive thymoma who was successfully treated with systemic chemotherapy, surgical resection and irradiation. The patient has been in continuous remission for 6 years without radiographic evidence of tumour recurrence.</p>\r\n', 'Imported with node_import.', 1349968611, 1),
(446, 449, 1, 'Oncocytic Schneiderian Papilloma : A Rare Sinonasal Papilloma Removed via Endoscopic Medial Maxillectomy', '<p>\r\n	There are three types of papilloma which arises from the Schneiderian membrane namely inverted, fungiform and oncocytic. Oncocytic papilloma is the rarest type and occurs predominantly in the older age group. Clinically, its behaviour is similar to inverted papilloma in having both local recurrence and associated malignancy. We report a rare case of oncocytic papilloma arising from the maxillary sinus, extending into the nasal cavity and nasopharynx, in a 78-year-old lady. Despite the longstanding history there was no evidence of associated malignancy in this lesion. An endoscopic medial maxillectomy was successfully utilized for approach and complete tumour excision. In conclusion, oncocytic papilloma should be considered as a differential diagnosis in patients presenting with unilateral polypoid nasal lesions especially in the elderly. In addition, endoscopic medial maxillectomy can provide an adequate approach for complete tumour removal.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349969057, 1),
(447, 450, 1, 'Ascariasis among the Orang Asli Children at Pos Lenjang, Pahang, Malaysia', '<p>\r\n	A cross-sectional study was carried out in 2006 to determine the prevalence of <em>Ascaris lumbricoides</em> infection among Orang Asli (aborigines) children in Pos Lenjang, Pahang. A total of 71 faecal samples were collected from the children (40 girls and 31 boys) aged between 1-12 years. The samples were examined for the presence of the ova of <em>A. lumbricoides</em> using direct faecal smear and formalin-ether concentration techniques.&nbsp; The results revealed that the overall prevalence of <em>A. lumbricoides</em> infection was 42.3%. The prevalence of <em>A. lumbricoides</em> infection was found to be slightly higher in males (45.2%) compared to females (40%) (p&gt;0.05). The school-aged children showed higher prevalence of infection (48.6%) as compared to the preschool children (35.3%) (p&lt;0.05). The high prevalence of <em>A. lumbricoides</em> infection in these Orang Asli children was associated with low socioeconomic status, poor environmental sanitation and personal hygiene. In the 30 samples positive for <em>Ascaris </em>ova, a detection rate of 100% was recorded using the&nbsp; formalin-ether concentration technique as compared to 46.7% with the direct smear technique (p&lt;0.05). Thus, it is recommended that both techniques (the direct smear and concentration techniques) should be performed routinely for a more accurate diagnosis.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349969702, 1),
(448, 451, 1, 'Prevalence of Human Papillomavirus Genotypes in Preinvasive and Invasive Cervical Cancer-A UKM Study', '<p>\r\n	A cross sectional study was done to determine the prevalence and distribution of human papillomavirus (HPV) genotypes in pre-invasive (cervical intraepithelial neoplasia, grade 3 or CIN 3) and invasive cervical cancer (ICC), in Universiti Kebangsaan Malaysia Medical Centre (UKMMC). A total of 80 paraffin-embedded tumour tissue blocks (20 CIN 3, 60 invasive cancers) between 1999 to 2007 were retrieved from the archives of the Department of Pathology.&nbsp; Patient&rsquo;s medical records were obtained from the Medical Records Office. Among invasive cancers (n=60), squamous cell carcinoma (SCC) account for 75% and adenocarcinoma 25%. The mean age of cases studied was 52.0 &plusmn; 12.2 years and Chinese was the predominant ethnicity (66.3%). Twelve HPV genotypes were identified, namely, HPV 16, 33, 18, 39, 52, 45, 58, 59, 31, 35, 6 and 11. The prevalence of HPV was 92.5% with types 16 being the most common (73.8%), followed by types 33 (30%) and 18 (22.5%). A total of 31 cases (38.8%) showed single HPV genotype, while 43 (53.8%) had multiple HPV (two genotypes or more) genotypes.&nbsp; In ICC, HPV 16, followed by types 33, 18, 52 and 39 were the top five common HPV genotypes detected. High prevalence of HPV and multiple HPV infections were major findings among patients with pre-invasive and invasive cervical cancer.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349988856, 1),
(449, 452, 1, 'Quality Of Life amongst Post Coronary Artery Bypass Patients at the National Heart Institute, Malaysia', '<p>\r\n	Coronary Artery Bypass Grafting (CABG) surgery has increased the survival rate for in&shy;dividuals suffering from myocardial infarction or coronary ischemia.&nbsp; The present study aimed to examine the perception of quality of life amongst post CABG patients at the National Heart Institute Malaysia. A total of 69 post CABG patients, were studied using the Medical Outcomes Short Form 36 (SF-36). The findings showed that post CABG respondents possessed good level of quality of life (SF-36) physically and mentally. There were significant differences between the role physical in the physical functioning do&shy;mains of quality of life with gender (t=0.286, p&lt;0.05). Duration of physi&shy;cal exercises of more than 20 minutes have positive effects on both physical (t=2.738, p&lt;0.05) and mental components (t=7.326, p&lt;0.05). The level of income influenced both the physical component (F=3.100, p&lt;0.05), and mental component (F=4.272, p&lt;0.05). Post CABG patients were found to have high quality of life post operatively. They were able to make comprehensive lifestyle changes through frequent physical exercises to accommodate their needs.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349989905, 1),
(450, 453, 1, 'Retrospective Review of Facial Nerve Schwannomas: The Universiti Kebangsaan Malaysia Medical Centre Experience', '<p>\r\n	Facial nerve schwannoma is a rare slow growing benign tumour which arises from the Schwann cell of the neurilemma. A retrospective review of 6 patients who had been diagnosed with facial nerve schwannoma between 1998 and 2008 was conducted. There was equal distribution of male and female patients. The mean age was 42 years (range 19 to 66 years). The tumour originated in the internal auditory canal (2 pa&shy;tients), intra-temporal (3 patients) and intraparotid (1 patient) segments of the facial nerve.&nbsp; All tumours were successfully removed and facial nerve continuity was pre&shy;served in 2 cases. The presenting symptoms of facial nerve schwannoma are non specific and dependent on the site of tumour origin. It is a great mimicker of other le&shy;sions that can present at the same location. The surgeon should have a high index of suspicion when patients present with progressive facial nerve palsy. Patients should always be counselled regarding risk of facial paralysis because the diagnosis of facial nerve schwannoma is often confirmed intra-operatively.</p>\r\n', '<p>\r', 'Imported with node_import.', 1349990433, 1),
(451, 454, 1, 'Retrospective Review of the Adjunctive Use of Pre-Operative Ranibizumab “LUCENTISTM” in the Surgical Management of Diabetic Retinopathy in a Tertiary Referral Hospital in Malaysia', '<p>\r\n	A retrospective case series review was conducted to determine the pre-operative role and safety of pre-operative adjunctive anti-vascular endothelial growth factor (anti-VEGF) agent ranibizumab &ldquo;LUCENTIS<sup>TM</sup>&rdquo; in patients with diabetic retinopathy requiring vitrectomy. The study involved twenty consecutive eyes of sixteen patients (age range: 46-72 years; mean 57.5 years) which received intravitreal injection of 0.5 - 1 mg of ranibizumab 3 to 8 days (mean 4.4 days) prior to vitrectomy for diabetic retinopathy. There were no local or systemic post-injection complications. Indications for vitrectomy were retinal detachment (RD) [n=11; 3 combined tractional (TRD) - rhegmatogenous RD (RRD), 8 TRD], TRD with vitreous haemorrhage (VH) (n=3) ,VH (n=8) and vitreo-macular traction syndrome (n=1). Inclusion criteria include all consecutive eyes of diabetic patients requiring vitrectomy receiving a first pre-operative injection of anti-VEGF. Pre-operative visual acuity (VA) ranged from 6/36 to light perception. All eyes had minimal to moderate intraoperative bleeding.&nbsp; Post-operative VH in eyes without tamponade or gas tamponade was nil (n=1), mild (n=13) or moderate (n=1). Silicone filled eyes had nil (n=1), moderate (n=3) or severe haemorrhages (n=1). Post-operative VA was unchanged (n=2) (10%), improved (n=14) (70%) or worsened (n=4). VA was 2/60 or better (n=15) to no light perception (n=1). Two eyes achieved 6/12 or better vision (10%). Ten eyes (50%) had 6/36 or better vision. In conclusion, pre-operative intravitreal ranibizumab is safe and useful in diabetic vitrectomy and appears to help with perioperative bleeding leading to improvement in vision.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350030375, 1),
(452, 455, 1, 'Congenital or Acquired? A Case Report of Extensive Congenital Cholesteatoma', '<p>\r\n	Congenital cholesteatoma (CC) is rare clinical condition and It is known as a pearly white mass medial to an intact tympanic membrane. In this case report, we describe a 6-year-old boy who presented with a one year history of left ear discomfort, reduced hearing, followed by ear discharge two months prior to admission. Otoscopic examination revealed a large attic perforation. Radiological investigation showed extensive cholesteatoma in the mastoid, mesotympanum and epitympanum with destruction of the ossicles including the stapes footplate. He underwent radical mastoidectomy in view of extensive cholesteatoma with tympanoplasty type V. In this case report, the classification, treatment and theories related to CC are discussed.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350031309, 1),
(453, 456, 1, 'Cri-du-chat syndrome: Application of array CGH in diagnostic evaluation', '<p>\r\n	The human genome contains many submicroscopic copy number variations which includes deletions, duplications and insertions. Although conventional karyotyping remains an important diagnostic tool in evaluating a dysmorphic patient with mental retardation, molecular diagnostic technology such as array comparative genomic hybridization (aCGH) has proven to be sensitive and reliable in detecting these submicroscopic anomalies. A 3 month-old infant with dysmorphic facies, microcephaly and global developmental delay was referred for genetic evaluation. Preliminary karyotyping which was confounded by the quality of metaphase spread was normal; however, aCGH detected a 30.6Mb deletion from 5p15.33-p13.3. This case illustrates the usefulness of aCGH as an adjunctive investigative tool for detecting chromosomal imbalances.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350032832, 1),
(454, 457, 1, 'Primary Thyroid Lymphoma: Two different presentations of a rare disease', '<p>\r\n	Primary thyroid lymphoma is a rare disorder accounting for about 2% of all malignant lymphomas and less than 5% of thyroid malignancies. It is an aggressive disease with poor outcome. The majority of thyroid lymphomas are non-Hodgkin lymphomas of B-cell origin. Majority of cases occur in women in the sixth decade. We report two cases of pri&shy;mary thyroid lymphoma and highlight the clinical issues and challenges posed by this rare disease. Both cases presented with respiratory obstructive symptoms that required surgical intervention. The optimal management for a primary thyroid lym&shy;phoma be it chemotherapy, radiotherapy, surgery or monoclonal antibodies is still de&shy;batable. The role for surgery has evolved through the years but its importance in emergency situations should not be overlooked. Both our patients had to undergo sur&shy;gery but only one patient received additional chemotherapy and radiotherapy. These two case reports illustrated the difficulties in managing this rare disorder.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350035413, 1),
(455, 458, 1, 'Effects of Phytosterol Supplementation on Lipid Peroxidation Induced by Carbon Tetrachloride in a Rat Model', '<p>\r\n	&nbsp;Phytosterols&nbsp; are&nbsp; plant&nbsp; sterols&nbsp; with&nbsp; a&nbsp; chemical&nbsp; structure&nbsp; similar&nbsp; to&nbsp; cholesterol.&nbsp; It&nbsp; has anti-cholesterol,&nbsp; anti-cancer&nbsp; and&nbsp; anti-oxidant&nbsp; properties&nbsp; which&nbsp; are&nbsp; probably&nbsp; mediated by suppression of lipid peroxidation. However, there are limited studies on the effects of phytosterols on lipid peroxidation. The aim of this study is to determine the effects of phytosterols on plasma and tissue malondialdehyde (MDA) of rats exposed to carbon tetrachloride.&nbsp; The&nbsp; rats&nbsp; were&nbsp; divided&nbsp; into&nbsp; four&nbsp; groups&nbsp; of&nbsp; normal&nbsp; control&nbsp; (NC),&nbsp; carbon tetrachloride&nbsp; (CCl4),&nbsp; phytosterol&nbsp; (P)&nbsp; and&nbsp; phytosterol+carbon&nbsp; tetrachloride&nbsp; (P+CCl4). The&nbsp; P and P+CCl4&nbsp; groups&nbsp; were&nbsp; pretreated&nbsp; with&nbsp; subcutaneous&nbsp; phytosterol&nbsp; at&nbsp; 140 mg/kg once weekly for 5 weeks while the NC and CCl4&nbsp; groups only received olive oil (vehicle). A single oral dose of carbon tetrachloride was then given to rats in the CCl4 and&nbsp; P+CCl4&nbsp; groups&nbsp; to induce&nbsp; lipid&nbsp; peroxidation.&nbsp; After&nbsp; 24 hours,&nbsp; all&nbsp; the&nbsp; rats&nbsp; were sacrificed and the plasma and tissue MDA were measured. Our results showed carbon tetrachloride had caused significant elevations of the plasma and hepatic MDA of the CCl4 group compared to the NC group. Phytosterol pretreatment (P+CCl4 group) were able to prevent the MDA elevations. Phytosterols treatments in normal rats (P group) were&nbsp; found&nbsp; to&nbsp; reduce&nbsp; the&nbsp; hepatic&nbsp; MDA&nbsp; level.&nbsp; The&nbsp; conclusion&nbsp; of&nbsp; this&nbsp; study&nbsp; was&nbsp; that phytosterols&nbsp; are&nbsp; effective&nbsp; suppressor&nbsp; of&nbsp; plasma&nbsp; and&nbsp; hepatic&nbsp; lipid&nbsp; peroxidation.&nbsp; They have&nbsp; potential&nbsp; as&nbsp; supplements&nbsp; to&nbsp; further&nbsp; reduce&nbsp; lipid&nbsp; peroxidation&nbsp; in&nbsp; healthy individuals.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350189866, 1),
(456, 459, 1, 'Genetic  Markers  in  Breast  Cancer- How  Far  Have  We Come from BRCA1?', '<p>\r\n	Breast cancer is the most common malignancy that develops in women worldwide, its incidence&nbsp; continues&nbsp; to&nbsp; rise&nbsp; and&nbsp; it&nbsp; is&nbsp; responsible&nbsp; for&nbsp; the&nbsp; highest&nbsp; death&nbsp; rates.&nbsp; Breast cancer&nbsp; can&nbsp; be&nbsp; classified&nbsp; as&nbsp; sporadic&nbsp; or&nbsp; familial&nbsp; -&nbsp; the&nbsp; strongest&nbsp; risk&nbsp; factor&nbsp; today&nbsp; is&nbsp; a family&nbsp; history.&nbsp; Germline&nbsp; mutations&nbsp; in&nbsp; high-penetrance&nbsp; breast&nbsp; cancer&nbsp; susceptibility genes BRCA1 and BRCA2 have been strongly implicated in the genetic predisposition of approximately 20% of familial breast cancers. Although BRCA1 and BRCA2 do not account&nbsp; for&nbsp; all&nbsp; familial&nbsp; breast&nbsp; cancers,&nbsp; there&nbsp; are&nbsp; currently&nbsp; no&nbsp; other&nbsp; genes&nbsp; that&nbsp; have been identified which segregate with familial breast cancer as strongly. Despite large-scale&nbsp; attempts&nbsp; to&nbsp; identify&nbsp; genetic&nbsp; risk&nbsp; factors&nbsp; associated&nbsp; with&nbsp; breast&nbsp; cancer,&nbsp; the&nbsp; variants identified through genome-wide association studies (GWAS), only confer a modest increase in risk of breast cancer and at present lack clinical utility. This review will discuss&nbsp; the&nbsp; known&nbsp; genetic&nbsp; risk&nbsp; factors&nbsp; for&nbsp; developing&nbsp; breast&nbsp; cancer&nbsp; and&nbsp; how&nbsp; far&nbsp; the field has progressed since the identification of BRCA1.</p>\r\n', '<p>\r', 'Imported with node_import.', 1352138019, 1),
(457, 460, 1, 'Elevated Expression of CD151 Gene in Estrogen Receptor and Progesterone Receptor Positive Breast Carcinoma', '<p>\r\n	The&nbsp; tetraspanin&nbsp; gene,&nbsp; CD151&nbsp; is&nbsp; involved&nbsp; in various&nbsp; tumour&nbsp; cell&nbsp; progression&nbsp; and metastasis. Its expression is increased in high grade, estrogen receptor negative and c-erbB-2&nbsp; positive&nbsp; breast&nbsp; cancer.&nbsp; However,&nbsp; the&nbsp; biological&nbsp; function&nbsp; and&nbsp; expression phenotype among different tumour status, estrogen receptor (ER) status, progesterone receptor (PR) status and c-erbB-2 expression in multi-ethnic Malaysian breast cancer patients&nbsp; has&nbsp; not been&nbsp; well&nbsp; investigated.&nbsp; We&nbsp; used&nbsp; quantitative&nbsp; real-time&nbsp; reverse transcriptase&nbsp; polymerase&nbsp; chain&nbsp; reaction&nbsp; (qRT-PCR)&nbsp; to&nbsp; measure&nbsp; the&nbsp; CD151&nbsp; gene expression&nbsp; in&nbsp; 45&nbsp; breast&nbsp; cancers.&nbsp; Our&nbsp; preliminary&nbsp; results&nbsp; revealed&nbsp; that&nbsp; CD151 expression is significantly higher in ER positive and PR positive breast cancers at 95% and&nbsp; 99%&nbsp; confidence&nbsp; intervals,&nbsp; respectively.&nbsp; In&nbsp; contrast,&nbsp; there&nbsp; is&nbsp; no&nbsp; significant correlation between CD151 expression and tumour grades or c-erbB-2 status at 95% confidence&nbsp; interval&nbsp; level.&nbsp; Our&nbsp; preliminary&nbsp; findings&nbsp; suggested&nbsp; that&nbsp; CD151&nbsp; may&nbsp; be involved in the estrogen responsive pathways. CD151 could be a potential prognostic marker&nbsp; and&nbsp; therapeutic&nbsp; target&nbsp; in&nbsp; the&nbsp; treatment&nbsp; of&nbsp; estrogen&nbsp; dependent&nbsp; breast&nbsp; cancer patients.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350215458, 1),
(458, 461, 1, 'Level of Recall between Verbal and Written Information about Orthodontic Treatment', '<p>\r\n	A cross-sectional study was undertaken to compare patients&rsquo; level of recall on verbal and written information about orthodontic treatment.&nbsp; Participants were the patients and par- ents attending the orthodontic screening clinic, Universiti Kebangsaan Malaysia (UKM). Participation&nbsp; was voluntary.&nbsp; Participants&nbsp; were&nbsp; placed into two&nbsp; groups.&nbsp; One group was given only written information in the form of an information leaflet, while another group was given only verbal information. The information content comprising of 13 items was based&nbsp; on&nbsp; the&nbsp; Patient&nbsp; Information&nbsp; Leaflet&nbsp; published&nbsp; by&nbsp; the&nbsp; British&nbsp; Orthodontic&nbsp; Society. Both&nbsp; groups&nbsp; received&nbsp; similar&nbsp; content&nbsp; of&nbsp; information&nbsp; in&nbsp; their&nbsp; preferred&nbsp; languages.&nbsp; Self administered&nbsp; close-ended&nbsp; questionnaire&nbsp; forms&nbsp; were&nbsp; given&nbsp; to&nbsp; the&nbsp; subjects&nbsp; after&nbsp; 15&nbsp; mi- nutes to assess the level of recall. The answers from both groups were compared. Data collected&nbsp; was&nbsp; analyzed&nbsp; using&nbsp; SPSS&nbsp; version&nbsp; 15.0.&nbsp; Chi-square&nbsp; test&nbsp; (p=0.05)&nbsp; was&nbsp; con- ducted to determine the effect of the method of information on the percentage of recall. A total&nbsp; of&nbsp; 79&nbsp; subjects&nbsp; volunteered&nbsp; to&nbsp; participate&nbsp; in&nbsp; the&nbsp; study.&nbsp; There&nbsp; were&nbsp; more&nbsp; females than males. The majority were found to have achieved secondary level formal education. Comparison of each question item showed no significant difference between those who were provided with verbal or written information. Both groups showed a high proportion of correct responses. In conclusion, there was no difference in the patients&rsquo; level of recall whether the orthodontic patient was given verbal or written information.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350241343, 1),
(459, 462, 1, 'Susceptibility to Smoking as a Predictor of Smoking Initiation among Adolescents-A Longitudinal Study in Kota Tinggi District, Malaysia', '<p>\r\n	Susceptibility&nbsp; to&nbsp; smoking,&nbsp; which&nbsp; is&nbsp; defined&nbsp; as&nbsp; a lack&nbsp; of&nbsp; cognitive&nbsp; commitment&nbsp; not to smoke in the future, has been shown to be a predictor for adolescent smoking initiation&nbsp; in&nbsp; developed&nbsp; countries.&nbsp; This&nbsp; study&nbsp; aims&nbsp; to evaluate&nbsp; the utility&nbsp; of&nbsp; a susceptibility-to- smoke measure as a predictor of smoking initiation among adolescents in Kota Tinggi district,&nbsp; Johor&nbsp; Malaysia.&nbsp; Susceptibility&nbsp; to&nbsp; smoking&nbsp; was&nbsp; evaluated&nbsp; among&nbsp; 1763 adolescents at baseline using a two-item construct. At follow-up 12 months later, 1288 of the 1763 adolescents (73.1%) responded. Results&nbsp; showed 14.9% (n=188/1260) of the&nbsp; respondents&nbsp; were&nbsp; susceptible&nbsp; to&nbsp; smoking&nbsp; at&nbsp; baseline.&nbsp; Among&nbsp; the&nbsp; susceptible adolescents, 31.9% initiated smoking after one year. Respondents who were identified as susceptible to smoking by the measure were 3.7 times (95%; CI: 2.17- 6.30) more likely&nbsp; to&nbsp; initiate smoking compared to non-susceptible respondents&nbsp; after adjusting for gender,&nbsp; school&nbsp; locality,&nbsp; percentage&nbsp; of&nbsp; friends&nbsp; who&nbsp; smoke,&nbsp; father&nbsp; smoking,&nbsp; parental acceptance&nbsp; of&nbsp; smoking,&nbsp; and&nbsp; belief&nbsp; in&nbsp; the&nbsp; positive&nbsp; and&nbsp; negative&nbsp; consequences&nbsp; of smoking.&nbsp; The&nbsp; findings&nbsp; suggest&nbsp; that&nbsp; the&nbsp; susceptibility&nbsp; measure&nbsp; is&nbsp; a&nbsp; reliable&nbsp; predictor and can be used as a screening tool to identify adolescents who are at risk of initiating smoking.&nbsp;</p>\r\n', '<p>\r', 'Imported with node_import.', 1350243336, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(460, 463, 1, 'SyBr Green Real-Time PCR is a Useful Method in Screening for High Risk Human Papilloma Virus Types 16 and 18 in Neoplastic Cervical Cancer', '<p>\r\n	Human&nbsp; papillomavirus&nbsp; (HPV)&nbsp; plays&nbsp; an&nbsp; important&nbsp; role&nbsp; in&nbsp; the&nbsp; pathogenesis&nbsp; of&nbsp; cervical cancer. HPV has been found in 99.7% of cervical cancers worldwide. In Malaysia, it is the second most common cancer among women in all major ethnic groups. The main purpose of this study was to establish the method of SyBrGreen Real-Time PCR and apply it for identification of multiple infections of the two high risk HPV subtypes. In this study,&nbsp; 57&nbsp; positive&nbsp; samples&nbsp; for&nbsp; HPV&nbsp; 16&nbsp; and&nbsp; HPV&nbsp; 18&nbsp; were&nbsp; used&nbsp; to&nbsp; establish&nbsp; a&nbsp; simple and sensitive method to detect and identify HPV infection in the cervical neoplasia at different stages of the disease by using real-time ABICycler SyBrGreen 1 technology. The results showed 67 HPV genomes in 57 samples. HPV 16 genome was detected in 55/67&nbsp; (82%)&nbsp; cases&nbsp; while&nbsp; HPV&nbsp; 18&nbsp; was&nbsp; detected&nbsp; in&nbsp; 8/67&nbsp; (12%)&nbsp; cases&nbsp; with&nbsp; 4&nbsp; cases showing&nbsp; multiple infections of HPV 16 and HPV&nbsp; 18.&nbsp; HPV 16&nbsp; was&nbsp; the&nbsp; most prevalent followed&nbsp; by&nbsp; HPV&nbsp; 18.&nbsp; Using&nbsp; SyBr&nbsp; Green&nbsp; Real-Time&nbsp; PCR&nbsp; techniques,&nbsp; the&nbsp; results showed that DNA melting curve for HPV 16 had a peak around 80.2&ordm;C and Ct value of 20 cycles whereas the DNA melting curve for HPV 18 around 79.2&ordm;C and Ct&nbsp; value of 22 cycles. In conclusion, a SyBr Green&nbsp; Real-Time PCR method has the potential for clinical&nbsp; usage&nbsp; in&nbsp; detection&nbsp; and&nbsp; identification&nbsp; of&nbsp; HPV&nbsp; infection&nbsp; in&nbsp; cervical&nbsp; neoplasia&nbsp; at different stages of the disease.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350269508, 1),
(461, 464, 1, 'Effect of Higher Centrifugation Speed and Shortened Centrifugation Time on Prothrombin and Activated Thromboplastin Time', '<p>\r\n	Centrifugation of blood samples to produce platelet-poor plasma is one of the important steps for coagulation testing. Reduction of the time required for specimen processing without affecting quality of results should be ideal for tests which require immediate results. Centrifugation of platelet-poor plasma (3580 rpm) for 15 minutes performed for routine coagulation tests would prolong the turn-around time for an urgent test (30 minutes). This study was done to determine the effect of reducing centrifugation time for routine coagulation tests in order to meet the turn-around time (TAT) for urgent tests. Seventy-nine blood samples sent for routine coagulation tests, were assayed for prothrombin time (PT), activated partial thromboplastin time (APTT), fibrinogen level and platelet counts, using two different centrifugation speed for plasma preparation: centrifugation at 3580 rpm for 15 minutes and rapid centrifugation at 4000 rpm for five minutes. Paired sample t-test showed that there was a significant difference in the platelet count between the two groups (p=0.001). However, there was no significant difference in the normal APTT (p=0.16), abnormal APTT (p=0.80), abnormal PT (p=0.43) and the results of fibrinogen levels (p=0.36). In conclusion, rapid centrifugation at 4000 rpm for five minutes does not modify results of routine coagulation tests (PT, APTT and fibrinogen). It would be beneficial in providing rapid results for urgent coagulation tests.</p>\r\n', '<p>\r', 'Imported with node_import.', 1350324277, 1),
(462, 465, 1, 'Recurrent Epididymo-orchitis: A Clinical Presentation of Posterior Urethral Valve in a Child with Down Syndrome', '<p>\r\n	Posterior urethral valve (PUV) commonly presents during infancy. We report a case of a 5-year-old boy with Down syndrome who was diagnosed to have PUV in late childhood. He presented with recurrent left epididymo-orchitis, an uncommon but recognized com-plication of PUV. Our aim is to discuss this rare and delayed presentation of PUV in par-ticular amongst children with Down syndrome, method of diagnosis and complication of the disease if left untreated.</p>\r\n', '<p>\r\n	Posterior urethral valve (PUV) commonly presents during infancy. We report a case of a 5-year-old boy with Down syndrome who was diagnosed to have PUV in late childhood. He presented with recurrent left epididymo-orchitis, an uncommon but recognized com-plication of PUV. Our aim is to discuss this rare and delayed presentation of PUV in par-ticular amongst children with Down syndrome, method of diagnosis and complication of the disease if left untreated.</p>\r\n', 'Imported with node_import.', 1350350836, 1),
(463, 466, 1, 'Determinants of Glucose Control among Elderly Diabetics in Kulim', '<p class="rtejustify">\r\n	Globally, the prevalence of diabetes mellitus and the average life expectancy is on the rise. As diabetes mellitus is commonly associated with old age, it is very important to ensure&nbsp; good&nbsp; glucose&nbsp; control&nbsp; to&nbsp; reduce&nbsp; complications&nbsp; and&nbsp; improve&nbsp; quality&nbsp; of&nbsp; life.&nbsp; A cross sectional study was conducted among elderly diabetics in Kulim with the aim to determine the prevalence of glucose control and its associated factors.&nbsp; Three hundred and twenty nine respondents were chosen through stratified random sampling in all the seven&nbsp; health&nbsp; clinics&nbsp; in&nbsp; Kulim.&nbsp; Respondents&nbsp; were&nbsp; interviewed&nbsp; personally&nbsp; using&nbsp; a structured&nbsp; questionnaire.&nbsp; HbA1c&nbsp; level&nbsp; of&nbsp; 6.5%&nbsp; and&nbsp; below&nbsp; was&nbsp; considered&nbsp; as&nbsp; good glucose control. Prevalence of good glucose control was 22.5%. Being male (Adjusted prevalence odds ratio, APOR = 1.75, 95% CI: 1.02-3.00), age of 70 years and above (APOR = 2.48, 95% CI: 1.40-4.39) and duration of diabetes less than five years (APOR =&nbsp; 2.10,&nbsp; 95%&nbsp; CI:1.21-3.64&nbsp; )&nbsp; were&nbsp; found&nbsp; to&nbsp; have&nbsp; significant&nbsp; association&nbsp; with&nbsp; good glucose&nbsp; control.&nbsp; As&nbsp; a&nbsp; conclusion,&nbsp; this&nbsp; study&nbsp; showed&nbsp; that&nbsp; the&nbsp; low&nbsp; prevalence&nbsp; of&nbsp; good glucose control was determined by gender, age and duration of illness.</p>\r\n', '<p class="rtejustify">\r', 'Imported with node_import.', 1354777000, 1),
(464, 467, 1, 'Perceived Stress Related to Nursing Education and Its Influence on Nursing Students'' Academic and Clinical Performance.', '<p class="rtejustify">\r\n	Stress has a negative effect on student nurses well-being and can impede learning or motivate them and is conducive to learning. This study examined the perceived stress and factors that influenced daily students&rsquo; life among both the Diploma and Bachelor of Nursing students. A total of 241 nursing students were involved in this research project. Findings of this study indicated that junior nursing students (&lt;21 years) of both cohorts perceived significantly more stress than older (&ge; 21 years) nursing students. Second year students of the Diploma group claimed being more stressed than their peers (1st and 3rd years). Personal problems were the main issue of concern among the second year Diploma students. Workload of nursing education and fear of failing the nursing programme were the major stressors among the first year nursing students of the Bachelor cohort as compared to their seniors. A mixture of negatively and positively significant correlations was observed between academic work and clinical scores among the Diploma and Bachelor of Nursing students respectively. Both the Diploma and Bachelor of Nursing students&rsquo; stress did not influence their GPA scores. Negatively significant correlation was noted between stress and clinical scores among the Diploma of Nursing students. A negatively significant correlation was found between age and CGPA scores among the Bachelor cohort. Both male and female Diploma and Bachelor nursing students perceived no differences in stress related to their academic work. The findings of this study provide important information for nursing education at the institute examined in this study, and have implications for future research.</p>\r\n', '<p class="rtejustify">\r', 'Imported with node_import.', 1348685603, 1),
(465, 468, 1, 'Prevalence and Awareness of Lower Urinary Tract Symptoms among Males in the Outpatient Clinics of Universiti Kebangsaan Malaysia Medical Centre', '<p class="rtejustify">\r\n	This study aims to determine the prevalence of lower urinary tract symptoms (LUTS) and level of awareness&nbsp; among male outpatients in Universiti Kebangsaan Malaysia Medical Centre (UKMMC). A questionnaire consisting of demographic data, questions related to knowledge, attitude and practice on BPH and the International Prostate Symptom Score (IPSS) was used for this study. Uroflowmetry and bladder scan were used to evaluate the function of the urinary tract and severity of BPH. Urine dipstick was done for glycosuria, proteinuria and haematuria. A total of 220 respondents were surveyed. The prevalence of moderately and severely symptomatic LUTS was 42.7%. The most commonly reported LUTS were nocturia (78.2%), frequency&nbsp; (58.2%) and incomplete emptying (44.6%). The prevalence of glycosuria, proteinuria and haematuria were 23.6%, 11.4% and 1.8% respectively. There was a significant association between increasing age with the severity of LUTS (p=0.005). Out of 102 respondents with voided urine volume greater than 150 mL, there was a significant decrease in maximum (Q<sub>max</sub>) (p=0.039) and average (Q<sub>ave</sub>) urine flow rates with every 10 years increase of age (p=0.001). The majority of respondents (59.5%) have heard of BPH before. Over 78.2% of the respondents would seek medical attention if they have LUTS with 15.9% saying they would seek traditional treatment. In conclusion, the prevalence of LUTS was high and the level of awareness was satisfactory.</p>\r\n', '<p class="rtejustify">\r', 'Imported with node_import.', 1348717150, 2),
(467, 470, 1, 'Structured Periodic Rehabilitation Intervention Sessions To Stroke Patients. A Longitudinal Review', '<p>\r\n	Despite evidence showing that structured rehabilitation after stroke improves functional outcomes,&nbsp; providing&nbsp; seamless&nbsp; rehabilitation&nbsp; from&nbsp; hospital&nbsp; to&nbsp; community&nbsp; has&nbsp; been elusive.&nbsp; The&nbsp; service&nbsp; provided&nbsp; should&nbsp; be&nbsp; able&nbsp; to&nbsp; accommodate&nbsp; variable&nbsp; degree&nbsp; of impairments and needs of the stroke survivors.&nbsp; This study aimed to assess the outcome of&nbsp; a&nbsp; multidisciplinary-based&nbsp; outpatient&nbsp; rehabilitation&nbsp; service&nbsp; for&nbsp; stroke&nbsp; patients&nbsp; living&nbsp; in the&nbsp; community.&nbsp; Patients&nbsp; who&nbsp; were&nbsp; discharged&nbsp; from&nbsp; Universiti&nbsp; Kebangsaan&nbsp; Malaysia Medical&nbsp; Centre&nbsp; after&nbsp; an&nbsp; acute&nbsp; stroke&nbsp; were&nbsp; referred&nbsp; to&nbsp; the&nbsp; Combined&nbsp; Stroke Rehabilitation&nbsp; Clinic&nbsp; (CSRC).&nbsp; Post&nbsp; stroke&nbsp; patients&nbsp; were&nbsp; assigned&nbsp; individually&nbsp; designed programs which were problem based and used task specific activities to achieve desired goals. Patients were reviewed on a regular basis either up to completion of the 2 year- program, or are able to be discharged to the community, whichever is earlier. Modified Barthel Index (MBI) and Berg Balance Scores (BBS) were used for monitoring. A total of 68 patients were managed in CSRC for two years since its initiation, with mean age of 62.4 years (SD 12.4) with the mean duration of stroke when first reviewed in CSRC was 11.5 months (SD 11.9). Majority of patients (64.7%) received either two or three types of intervention. Both MBI and BBS demonstrated significant improvement over 12-months period&nbsp; (p&nbsp; value&nbsp; of&nbsp; 0.006&nbsp; and&nbsp; 0.017&nbsp; respectively).&nbsp; CSRC&nbsp; proved&nbsp; that&nbsp; structured rehabilitation intervention was beneficial in terms of functional status and improvement in balance to post-stroke patients.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348712889, 1),
(468, 471, 1, 'Amitraz Poisoning: A Case Report', '<p>\r\n	Amitraz&nbsp; poisoning&nbsp; is&nbsp; rare&nbsp; and&nbsp; there&nbsp; has&nbsp; been&nbsp; no&nbsp; reported&nbsp; cases&nbsp; in&nbsp; the&nbsp; Malaysian literature.&nbsp; Ingestion&nbsp; of&nbsp; this&nbsp; compound&nbsp; carries&nbsp; many&nbsp; life&nbsp; threatening&nbsp; side&nbsp; effects.&nbsp; We describe&nbsp; a&nbsp; case&nbsp; of&nbsp; Amitraz&nbsp; poisoning&nbsp; in&nbsp; an&nbsp; 18&nbsp; years&nbsp; old&nbsp; young&nbsp; adult.&nbsp; He&nbsp; developed hypotension, required ventilatory support and a day of intensive care unit stay. He had a quick recovery after he was treated symptomatically and was discharged well after 3 days.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348696721, 1),
(469, 472, 1, 'Apparent Factor IX Inhibitor', '<p>\r\n	The causes of an isolated prolonged activated partial thromboplastin time (APTT) with a normal prothrombin time (PT) are either a deficiency of clotting factors VIII, IX, XI or XII or the presence of an inhibitor. The inhibitor may be specific to an individual clotting factor or it may be a non-specific inhibitor like the lupus anticoagulant which has opposite therapeutic implications. We report a patient referred to our hospital for treatment that was previously diagnosed at another medical institution as an acquired factor IX inhibitor following an investigation for a prolonged APTT. On further testing this turned out to be a potent lupus anticoagulant which interfered with the phospholipid-dependent factor assays. The use of dilution studies, chromogenic assays and phospholipid neutralization can help differentiate these inhibitors. Great care must be taken in the interpretation of factor assays in the presence of lupus anti-<br />\r\n	coagulant to avoid misdiagnosis and inappropriate treatment.</p>\r\n', '<p>\r', 'Imported with node_import.', 1348698505, 1),
(472, 475, 1, '7th Malaysia Indonesia Brunei Medical Science Conference ''TOWARDS A HOLISTIC AND INTEGRATIVE APPROACH IN HEALTHCARE''', '', '', '', 1354548917, 1),
(473, 476, 1, '4 th  Malaysia Indonesia Brunei Medical Science Conference ''MEDICAL RESEARCH IN THE ‘OMICS’ ERA''', '', '', '', 1354775167, 1),
(474, 477, 64, 'Reviewer Agreement Form', '', '', '', 1357114689, 1),
(475, 478, 1, 'Reviewer Report Form', '', '', '', 1343918843, 1),
(476, 479, 1, 'Subscription', '<p>\r\n	Since October 2012, Medicine &amp; Health has become a fully online journal. All users can freely access abstracts and search journal content. Full text articles can be accessed after registration. Printed copies are also available upon request at an annual subscription of US$60 (oversea) and RM30 (Malaysia). All enquiries regarding subscription of Medicine &amp; Health should be addressed to:</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	The Editorial Office,<br />\r\n	Medicine &amp; Health,<br />\r\n	Department of Pathology,<br />\r\n	Faculty of Medicine,<br />\r\n	Universiti Kebangsaan Malaysia Medical Centre,<br />\r\n	Jalan Yaacob Latif, Bandar Tun Razak,<br />\r\n	56000 Cheras, Kuala Lumpur, Malaysia.<br />\r\n	Telephone No.: +60-3-9145 5372/7164/5815/5465<br />\r\n	Fax No.: +60-3-9145 6634<br />\r\n	Email address: <a href="mailto:editor@ppukm.ukm.edu.my" target="_blank">editor@ppukm.ukm.edu.my</a></p>\r\n', '<p>\r\n	Since October 2012, Medicine &amp; Health has become a fully online journal. All users can freely access abstracts and search journal content. Full text articles can be accessed after registration. Printed copies are also available upon request at an annual subscription of US$60 (oversea) and RM30 (Malaysia). All enquiries regarding subscription of Medicine &amp; Health should be addressed to:</p>\r\n<p>\r\n	&nbsp;</p>', '', 1351863198, 1),
(477, 480, 1, 'Jameela S', '', '', '', 1347820301, 0),
(478, 481, 1, 'Rozika P', '', '', '', 1347820302, 0),
(479, 482, 1, 'Rizalman J', '', '', '', 1347820302, 0),
(480, 483, 1, 'Visalachy P', '', '', '', 1347820302, 0),
(482, 485, 1, 'Choor CK', '', '', '', 1348628413, 0),
(483, 486, 1, 'Santha KN', '', '', '', 1348628413, 0),
(484, 487, 1, 'Gurbinder Kaur JS', '', '', '', 1348685332, 0),
(485, 488, 1, 'Hamidah H', '', '', '', 1348685332, 0),
(486, 489, 1, 'Blackman I', '', '', '', 1348685332, 0),
(487, 490, 1, 'Wotton K', '', '', '', 1348685332, 0),
(488, 491, 1, 'Belan I', '', '', '', 1348685332, 0),
(489, 492, 1, 'Christopher Ho CK', '', '', '', 1348686618, 0),
(490, 493, 1, 'Praveen S', '', '', '', 1348686618, 0),
(491, 494, 1, 'Goh EH', '', '', '', 1348686618, 0),
(492, 495, 1, 'Tan GH', '', '', '', 1348686618, 0),
(493, 496, 1, 'Badrulhisham B', '', '', '', 1348686618, 0),
(494, 497, 1, 'Zulkifli MZ', '', '', '', 1348686619, 0),
(495, 498, 1, 'Khoo HW', '', '', '', 1348686619, 0),
(496, 499, 1, 'Su XV', '', '', '', 1348686619, 0),
(497, 500, 1, 'Ng JY', '', '', '', 1348686619, 0),
(498, 501, 1, 'Chong LL', '', '', '', 1348686619, 0),
(499, 502, 1, 'Lim DW', '', '', '', 1348686619, 0),
(500, 503, 1, 'Chen ES', '', '', '', 1348686619, 0),
(501, 504, 1, 'Teoh SY', '', '', '', 1348686619, 0),
(502, 505, 1, 'Aziz NA', '', '', '', 1348687228, 0),
(503, 506, 1, 'Nashrah H', '', '', '', 1348687228, 0),
(504, 507, 1, 'Fadilah AZ', '', '', '', 1348687228, 0),
(505, 508, 1, 'Ali MF', '', '', '', 1348687228, 0),
(506, 509, 1, 'Zawawi S', '', '', '', 1348687228, 0),
(507, 510, 1, 'Katijah A ', '', '', '', 1348687228, 0),
(508, 511, 1, 'Leelavathi M', '', '', '', 1348739169, 0),
(509, 512, 1, 'Yasmin SAK', '', '', '', 1348739169, 0),
(510, 513, 1, 'Gomez PA', '', '', '', 1348739169, 0),
(511, 514, 1, 'Aznida FAA', '', '', '', 1348739169, 0),
(512, 515, 1, 'Reena RMZ', '', '', '', 1348740066, 0),
(513, 516, 1, 'Julia Munchar MJ', '', '', '', 1348740066, 0),
(514, 517, 1, 'Salwati S', '', '', '', 1348740066, 0),
(515, 518, 1, 'Zubaidah Z', '', '', '', 1348740067, 0),
(516, 519, 1, 'Sharifah NA', '<p>\r\n	Sharifah Noor Akmal</p>\r\n', '<p>\r\n	Sharifah Noor Akmal</p>\r\n', '', 1348896716, 1),
(517, 520, 1, 'Cheong SK', '', '', '', 1348740067, 0),
(518, 521, 1, 'Ho Siew Eng', '', '', '', 1348776445, 0),
(519, 522, 1, 'Gurbinder Kaur', '', '', '', 1348776445, 0),
(520, 523, 1, 'Syed Rozaidi Wafa', '', '', '', 1348776446, 0),
(521, 524, 1, 'Syed Zulkifli Syed Zakaria', '', '', '', 1348776447, 0),
(522, 525, 1, 'Razali Omar', '', '', '', 1348776447, 0),
(523, 526, 1, 'IM Sulaiman', '', '', '', 1348819512, 0),
(524, 527, 1, 'HS Lee', '', '', '', 1348819512, 0),
(525, 528, 1, 'S Balan', '', '', '', 1348819512, 0),
(526, 529, 1, 'MZ Jaafar', '', '', '', 1348819512, 0),
(527, 530, 1, 'Tong SF', '', '', '', 1348823670, 0),
(528, 531, 1, 'Chin GL', '', '', '', 1348823670, 0),
(529, 532, 1, 'Raja Lexshimi RG', '', '', '', 1348825422, 0),
(530, 533, 1, 'Raijah A. Rahim', '', '', '', 1348825422, 0),
(531, 534, 1, 'Phan CL', '', '', '', 1348826653, 0),
(532, 535, 1, 'Gregory A.R.A', '', '', '', 1348826653, 0),
(533, 536, 1, 'Ten SK', '', '', '', 1348826653, 0),
(534, 537, 1, 'Kamariah MN', '', '', '', 1348826653, 0),
(535, 538, 1, 'Thilagavathi S', '', '', '', 1348826653, 0),
(536, 539, 1, 'Maimonah R', '', '', '', 1348826653, 0),
(544, 547, 1, 'C-F Leong', '', '', '', 1348874973, 0),
(545, 548, 1, 'O Ainoon', '', '', '', 1348874973, 0),
(546, 549, 1, 'Fadilah SAW', '', '', '', 1348874973, 0),
(547, 550, 1, 'S-K Cheong', '', '', '', 1348874973, 0),
(548, 551, 1, 'Abdullah A', '', '', '', 1348887678, 0),
(549, 552, 1, 'Shaharudin MH', '', '', '', 1348887678, 0),
(550, 553, 1, 'Ami M', '', '', '', 1348887678, 0),
(551, 554, 1, 'Marhaban JA', '', '', '', 1348887678, 0),
(552, 555, 1, 'Awang MA', '', '', '', 1348887678, 0),
(553, 556, 1, 'Zulfiqar MA', '', '', '', 1348887678, 0),
(554, 557, 1, 'Othman BF', '', '', '', 1348887678, 0),
(555, 558, 1, 'Saim L', '', '', '', 1348887678, 0),
(556, 559, 1, 'Siti Aishah Md Ali', '', '', '', 1348890183, 0),
(557, 560, 1, 'Ilina Isahak', '', '', '', 1348890184, 0),
(558, 561, 1, 'Dahlan Sabil', '', '', '', 1348890184, 0),
(559, 562, 1, 'Fatimah Sahlan', '', '', '', 1348890184, 0),
(560, 563, 1, 'Lokman Saim', '', '', '', 1348890184, 0),
(561, 564, 1, 'Abdullah Sani Mohamed', '', '', '', 1348890184, 0),
(562, 565, 1, 'Nor Zarina ZA', '', '', '', 1348896866, 0),
(563, 566, 1, 'Wan Muhaizan WM', '', '', '', 1348900638, 0),
(564, 567, 1, 'Geok Chin Tan', '', '', '', 1348910484, 0),
(565, 568, 1, 'Mohd Sidik Shiran', '', '', '', 1348910484, 0),
(566, 569, 1, 'Manickam Swaminathan', '', '', '', 1348910484, 0),
(567, 570, 1, 'Md Ali Siti Aishah', '', '', '', 1348910484, 0),
(568, 571, 1, 'Poo Ling Ong', '', '', '', 1348910484, 0),
(569, 572, 1, 'Mohd Dali Ahmad Zailani Hatta', '', '', '', 1348910484, 0),
(570, 573, 1, 'Kok Howe Sen', '', '', '', 1348913143, 0),
(571, 574, 1, 'Tara Mary George', '', '', '', 1348913143, 0),
(572, 575, 1, 'Mae-Lynn Catherine Bastion', '', '', '', 1348913143, 0),
(573, 576, 1, 'Muhaya Hj Mohamad', '', '', '', 1348913143, 0),
(574, 577, 1, 'Santhna Letchmi', '', '', '', 1348930942, 0),
(575, 578, 1, 'Teoh Koi Hong', '', '', '', 1348930942, 0),
(576, 579, 1, 'Hanida Mokhtar', '', '', '', 1348930942, 0),
(577, 580, 1, 'Chee Kent Lim', '', '', '', 1348975629, 0),
(578, 581, 1, 'So Har Ton', '', '', '', 1348975629, 0),
(579, 582, 1, 'Ho SE', '', '', '', 1348976368, 0),
(580, 583, 1, 'Rohani M', '', '', '', 1348976369, 0),
(581, 584, 1, 'I Liliwati', '', '', '', 1348977043, 0),
(582, 585, 1, 'LKM Verna', '', '', '', 1348977043, 0),
(583, 586, 1, 'O Khairani', '', '', '', 1348977043, 0),
(584, 587, 1, 'Hatta Sidi', '', '', '', 1348979856, 0),
(585, 588, 1, 'Sharifah Ezat Wan Puteh', '', '', '', 1348979856, 0),
(586, 589, 1, ' Marhani Midin', '', '', '', 1348979856, 0),
(587, 590, 1, 'Norni Abdullah', '', '', '', 1348979856, 0),
(588, 591, 1, 'Nurismah MI', '', '', '', 1348986412, 0),
(589, 592, 1, ' Usama AE', '', '', '', 1348986413, 0),
(590, 593, 1, 'Rohaizak M', '', '', '', 1348986413, 0),
(591, 594, 1, 'Naqiyah I', '', '', '', 1348986413, 0),
(592, 595, 1, 'Jasmi A', '', '', '', 1348986413, 0),
(593, 596, 1, 'Adeeb N', '', '', '', 1348987799, 0),
(594, 597, 1, 'Nur Azurah AG', '', '', '', 1348987800, 0),
(595, 598, 1, ' Ong FB', '', '', '', 1348987800, 0),
(596, 599, 1, 'Seri SS', '', '', '', 1348987800, 0),
(597, 600, 1, 'Shamsuddin K', '', '', '', 1348987800, 0),
(598, 601, 1, 'Noor-Aini MY', '', '', '', 1348987800, 0),
(599, 602, 1, 'Khairani O', '', '', '', 1349017771, 0),
(600, 603, 1, ' Norazua R', '', '', '', 1349017771, 0),
(601, 604, 1, 'Zaiton A', '', '', '', 1349017771, 0),
(602, 605, 1, 'Santhna L.P', '', '', '', 1349044495, 0),
(603, 606, 1, 'Samsiah M', '', '', '', 1349044496, 0),
(604, 607, 1, 'Roshdinom R', '', '', '', 1349044496, 0),
(605, 608, 1, 'W.F. Swee', '', '', '', 1349059478, 0),
(606, 609, 1, 'Anza E', '', '', '', 1349059478, 0),
(607, 610, 1, 'Noor Hassim I', '', '', '', 1349059478, 0),
(608, 611, 1, 'S-A-W Fadilah', '', '', '', 1349060951, 0),
(609, 612, 1, 'V Shanty', '', '', '', 1349060952, 0),
(610, 613, 1, 'AS Goh', '', '', '', 1349060952, 0),
(611, 614, 1, 'Muhd Helmi Azmi', '', '', '', 1349061769, 0),
(612, 615, 1, 'Karis Misiran', '', '', '', 1349061769, 0),
(613, 616, 1, 'Ismail Mohd Saiboon', '', '', '', 1349065611, 0),
(614, 617, 1, 'Noraliza Mohd Ariffin', '', '', '', 1349065611, 0),
(615, 618, 1, 'Teodoro Javier Herbosa', '', '', '', 1349065611, 0),
(616, 619, 1, 'Ahmad Khaldun Ismail', '', '', '', 1349065611, 0),
(617, 620, 1, 'Nariman Singmamae', '', '', '', 1349065611, 0),
(618, 621, 1, 'Shamsuriani Md Jamal', '', '', '', 1349065611, 0),
(619, 622, 1, 'Azhana Hassan', '', '', '', 1349065611, 0),
(620, 623, 1, 'Santhna L. P', '', '', '', 1349066557, 0),
(621, 624, 1, 'Ruth P.R.D', '', '', '', 1349066557, 0),
(622, 625, 1, 'Tan GC', '', '', '', 1349067464, 0),
(623, 626, 1, 'Shiran MS', '', '', '', 1349067464, 0),
(624, 627, 1, 'Hatta AZ', '', '', '', 1349067464, 0),
(625, 628, 1, 'Ng HO', '', '', '', 1349067464, 0),
(626, 629, 1, 'Asma A', '', '', '', 1349068306, 0),
(627, 630, 1, 'Hazim MYS', '', '', '', 1349068309, 0),
(628, 631, 1, 'Marina MB', '', '', '', 1349068311, 0),
(629, 632, 1, 'Azizi AB', '', '', '', 1349068311, 0),
(630, 633, 1, 'Suraya A', '', '', '', 1349068311, 0),
(631, 634, 1, 'Norlaili MT', '', '', '', 1349068311, 0),
(632, 635, 1, 'Mazita A', '', '', '', 1349068312, 0),
(633, 636, 1, 'Zahiruddin', '', '', '', 1349068312, 0),
(634, 637, 1, 'Tan KC', '', '', '', 1349069350, 0),
(635, 638, 1, 'Chuah CY', '', '', '', 1349069350, 0),
(636, 639, 1, 'Fong KY', '', '', '', 1349069350, 0),
(637, 640, 1, 'Phang SL', '', '', '', 1349069350, 0),
(638, 641, 1, 'Nor Azlin MI', '', '', '', 1349069350, 0),
(639, 642, 1, 'Saadiah Tahir', '', '', '', 1349072640, 0),
(640, 643, 1, 'Md Nizam J', '', '', '', 1349072640, 0),
(641, 644, 1, 'Ainsah O', '', '', '', 1349083755, 0),
(642, 645, 1, 'Osman CB', '', '', '', 1349083755, 0),
(643, 646, 1, 'Gani AN', '', '', '', 1349084465, 0),
(644, 647, 1, 'Rozman Z', '', '', '', 1349084925, 0),
(645, 648, 1, 'Megat Shiraz MAR', '', '', '', 1349085493, 0),
(646, 649, 1, 'Siti Aishah MA', '', '', '', 1349085493, 0),
(647, 650, 1, 'Norazizah M', '', '', '', 1349085493, 0),
(648, 651, 1, 'N.A Aziz', '', '', '', 1349096676, 0),
(649, 652, 1, 'A.A Raymond', '', '', '', 1349096676, 0),
(650, 653, 1, 'Ismail Saiboon', '', '', '', 1349101798, 0),
(651, 654, 1, 'Bala Krishnan', '', '', '', 1349101798, 0),
(652, 655, 1, 'Siti Nooraini Ali', '', '', '', 1349101798, 0),
(653, 656, 1, 'Noorafindi Murad', '', '', '', 1349101798, 0),
(654, 657, 1, 'Audrey Pathnathan', '', '', '', 1349101798, 0),
(655, 658, 1, 'Choy Yin Choy', '', '', '', 1349101798, 0),
(656, 659, 1, 'Mohd Badli M', '', '', '', 1349102451, 0),
(657, 660, 1, 'Reena Rahayu Md Zin', '', '', '', 1349128297, 0),
(658, 661, 1, 'Zubaidah Zakaria', '', '', '', 1349128298, 0),
(659, 662, 1, 'Clarence Ko Ching Huat', '', '', '', 1349128298, 0),
(660, 663, 1, 'Siti Mariam Yusof', '', '', '', 1349128298, 0),
(661, 664, 1, 'Julia Mohd Idris', '', '', '', 1349128298, 0),
(662, 665, 1, 'Zarina Abdul Latif', '', '', '', 1349128298, 0),
(663, 666, 1, 'Wu Loo Ling', '', '', '', 1349128298, 0),
(664, 667, 1, 'Wong Ming', '', '', '', 1349128298, 0),
(665, 668, 1, 'Ay Eeng Tan', '', '', '', 1349130044, 0),
(666, 669, 1, 'Wai Kwong Choong', '', '', '', 1349130045, 0),
(667, 670, 1, 'Pooi Yan Leong', '', '', '', 1349130045, 0),
(668, 671, 1, 'Wei Mui Ng', '', '', '', 1349130045, 0),
(669, 672, 1, 'Soon Leong Yong', '', '', '', 1349130045, 0),
(670, 673, 1, 'Noor Hassim Ismail', '', '', '', 1349131624, 0),
(671, 674, 1, 'Rafiza Shaharuddin', '', '', '', 1349131624, 0),
(672, 675, 1, 'Sanisah Saidi', '', '', '', 1349139477, 0),
(673, 676, 1, 'Syed Zulkfli', '', '', '', 1349139477, 0),
(674, 677, 1, 'Abdus Salam', '', '', '', 1349140261, 0),
(675, 678, 1, 'Ahmad Faizal Mohd Perdaus', '', '', '', 1349140262, 0),
(676, 679, 1, 'Siti Harnida Md Isa', '', '', '', 1349140262, 0),
(677, 680, 1, 'Zulkifli Zainuddin', '', '', '', 1349140262, 0),
(678, 681, 1, 'Azian Abdul Latiff', '', '', '', 1349140262, 0),
(679, 682, 1, 'Ng Soon Pheng', '', '', '', 1349140262, 0),
(680, 683, 1, 'Zauyah Yusuf', '', '', '', 1349140262, 0),
(681, 684, 1, 'Ima Nirwana Soelaiman', '', '', '', 1349140262, 0),
(682, 685, 1, 'Nabishah Mohamad', '', '', '', 1349140262, 0),
(683, 686, 1, 'Norhayati Moktar', '', '', '', 1349140262, 0),
(684, 687, 1, 'Zahiah M', '', '', '', 1349140892, 0),
(685, 688, 1, 'Siti-Aishah M A', '', '', '', 1349141657, 0),
(686, 689, 1, 'Idrus M', '', '', '', 1349141658, 0),
(687, 690, 1, 'Rahimah R', '', '', '', 1349141658, 0),
(688, 691, 1, 'Salmi A', '', '', '', 1349141658, 0),
(689, 692, 1, 'Leong C.F', '', '', '', 1349141658, 0),
(690, 693, 1, 'Aniza I', '', '', '', 1349148631, 0),
(691, 694, 1, 'Saperi S', '', '', '', 1349148631, 0),
(692, 695, 1, 'Aljunid SM', '', '', '', 1349148631, 0),
(693, 696, 1, 'Nazrun AS', '', '', '', 1349149204, 0),
(694, 697, 1, 'Khairunnur A', '', '', '', 1349149204, 0),
(695, 698, 1, 'Norliza M', '', '', '', 1349149204, 0),
(696, 699, 1, 'Norazlina M', '', '', '', 1349149204, 0),
(697, 700, 1, 'Farahani ARS', '', '', '', 1349150349, 0),
(698, 701, 1, 'Zakiah J', '', '', '', 1349150349, 0),
(699, 702, 1, 'Abdul Rahman M', '', '', '', 1349150349, 0),
(700, 703, 1, 'Karsani SA', '', '', '', 1349150349, 0),
(701, 704, 1, 'Wan Ngah WZ', '', '', '', 1349150349, 0),
(702, 705, 1, 'Yasmin Anum MY', '', '', '', 1349151003, 0),
(703, 706, 1, 'Shahriza ZA', '', '', '', 1349151003, 0),
(704, 707, 1, 'Looi ML', '', '', '', 1349151003, 0),
(705, 708, 1, 'Shafina Hanim MH', '', '', '', 1349151003, 0),
(706, 709, 1, 'Harlianshah H', '', '', '', 1349151003, 0),
(707, 710, 1, 'Noor Aini AH', '', '', '', 1349151003, 0),
(708, 711, 1, 'Suzana M', '', '', '', 1349151003, 0),
(709, 712, 1, 'Wan Zurinah WN', '', '', '', 1349151003, 0),
(710, 713, 1, 'Mohd Said N', '', '', '', 1349153243, 0),
(711, 714, 1, 'Rogayah J', '', '', '', 1349153243, 0),
(712, 715, 1, 'Hafizah A', '', '', '', 1349153243, 0),
(713, 716, 1, 'Hayati Y', '', '', '', 1349153814, 0),
(714, 717, 1, 'Ting CK', '', '', '', 1349153814, 0),
(715, 718, 1, 'Oteh M', '', '', '', 1349153814, 0),
(716, 719, 1, 'Choy YC', '', '', '', 1349153814, 0),
(717, 720, 1, 'Tan AE', '', '', '', 1349154452, 0),
(718, 721, 1, 'Siti SA', '', '', '', 1349154452, 0),
(719, 722, 1, 'Wong HS', '', '', '', 1349159094, 0),
(720, 723, 1, 'William Chan YH', '', '', '', 1349159095, 0),
(721, 724, 1, 'Neeta KB', '', '', '', 1349159095, 0),
(722, 725, 1, 'Lum SG', '', '', '', 1349159095, 0),
(723, 726, 1, 'Seet KC', '', '', '', 1349159095, 0),
(724, 727, 1, 'Tan HL', '', '', '', 1349159095, 0),
(725, 728, 1, 'Naresh G', '', '', '', 1349159095, 0),
(726, 729, 1, 'Zainal Azizi A', '', '', '', 1349159095, 0),
(727, 730, 1, 'Ismail S', '', '', '', 1349159095, 0),
(728, 731, 1, 'Yusoff AN', '', '', '', 1349160295, 0),
(729, 732, 1, 'Abdul Hamid K', '', '', '', 1349160295, 0),
(730, 733, 1, ' Mohamad M', '', '', '', 1349160295, 0),
(731, 734, 1, 'Abd Hamid AI', '', '', '', 1349160295, 0),
(732, 735, 1, 'Amer Siddiq AN', '', '', '', 1349187659, 0),
(733, 736, 1, 'Radzniwan AR', '', '', '', 1349188499, 0),
(734, 737, 1, 'Azimah NM', '', '', '', 1349188499, 0),
(735, 738, 1, 'Zuhra H', '', '', '', 1349188499, 0),
(736, 739, 1, 'Musalmah M', '', '', '', 1349198675, 0),
(737, 740, 1, 'Rusdiah RJ', '', '', '', 1349198676, 0),
(738, 741, 1, 'Fazlinee Daud', '', '', '', 1349205610, 0),
(739, 742, 1, 'Nur Nadia MA', '', '', '', 1349214800, 0),
(740, 743, 1, 'Zahariah I', '', '', '', 1349214800, 0),
(741, 744, 1, 'Chua KH', '', '', '', 1349214800, 0),
(742, 745, 1, 'Zaleha MI', '', '', '', 1349218970, 0),
(743, 746, 1, 'Shamsul AS', '', '', '', 1349218971, 0),
(744, 747, 1, 'Suriawati G', '', '', '', 1349218971, 0),
(745, 748, 1, 'Rozainiee A', '', '', '', 1349219901, 0),
(746, 749, 1, 'Sharifa Ezat WP', '', '', '', 1349220683, 0),
(747, 750, 1, 'Azimatun NA', '', '', '', 1349220683, 0),
(748, 751, 1, 'Nik Jasmin NM', '', '', '', 1349220683, 0),
(749, 752, 1, 'Syed Aljunid', '', '', '', 1349220683, 0),
(750, 753, 1, 'Johan AJ', '', '', '', 1349221105, 0),
(751, 754, 1, 'Cheah FC', '', '', '', 1349221105, 0),
(752, 755, 5, 'Haerian BS', '', '', '', 1349236515, 0),
(753, 756, 5, 'Mohamed Z', '', '', '', 1349236516, 0),
(754, 757, 5, 'Mohamed EHM', '', '', '', 1349236516, 0),
(755, 758, 5, 'Lim KS', '', '', '', 1349236516, 0),
(756, 759, 5, 'Tan HJ', '', '', '', 1349236516, 0),
(757, 760, 5, 'Tan CT', '', '', '', 1349236516, 0),
(758, 761, 5, 'Raymond AA', '', '', '', 1349236516, 0),
(759, 762, 58, 'GOON JA', '', '', '', 1349237711, 0),
(760, 763, 58, 'NAFEESA NAJWA AK', '', '', '', 1349237711, 0),
(761, 764, 58, 'Tai Chi Exercise Modulates Physiological and Biochemical Parameters in Pre-Menopausal Women', 'Aging  is attributed  to accumulation of cellular damage due to oxidative stress. However,  adaptations to different  types  of regular exercise have been  shown to produce  beneficial  outcomes measurable at the physiological and cellular level even amongst the elderly. In this study, modulation of body fat, body mass index, heart  rate, blood pressure and blood biochemical profile  by Tai Chi exercise were  determined in healthy pre-menopausal  women aged 45 and above.  They performed the Tai Chi exercise led by an appointed instructor, twice a week for 1 hour every session. Parameters were compared with the baseline after 6 months. Fat percentage and heart rate decreased while high density lipoprotein, total protein, albumin  and calcium levels increased significantly.  Since the sedentary  controls had no significant changes, we concluded that regular Tai Chi exercise improve the health status of pre-menopausal women.', 'Aging  is attributed  to accumulation of cellular damage due to oxidative stress. However,  adaptations to different  types  of regular exercise have been  shown to produce  beneficial  outcomes measurable at the physiological and cellular level even amongst the elderly. In this study, modulation of body fat, body mass index, heart  rate, blood pressure and blood biochemical profile  by Tai Chi exercise were  determined in healthy pre-menopausal  women aged 45 and above.  They performed the Tai Chi exercise led by an appointed instructor, twice a week for 1 hour every session.', '', 1349237712, 1),
(762, 765, 1, 'Review of Tai Chi Exercise Modulates Physiological and Biochemical Parameters in Pre-Menopausal Women', '', '', '', 1349238518, 0),
(761, 766, 1, 'Tai Chi Exercise Modulates Physiological and Biochemical Parameters in Pre-Menopausal Women 2', '<p>\r\n	Aging is attributed to accumulation of cellular damage due to oxidative stress. however, adaptations to different types of regular exercise have been SHOWN to produce outcomes measurable at the physiological and cellular level even amongst the elderly. In this study, modulation of body fat, body mass index, heart rate, blood pressure and blood biochemical profile by Tai Chi exercise were determined in healthy pre-menopausal women aged 45 and above. They performed the Tai Chi exercise led by an appointed instructor, twice a week for 1 hour every session. Parameters were compared with the baseline after 6 months. Fat percentage and heart rate decreased while high density lipoprotein, total protein, albumin and calcium levels increased significantly. Since the sedentary controls had no significant changes, we concluded that regular Tai Chi exercise improve the health status of pre-menopausal women.</p>\r\n', '<p>\r', 'test', 1358329139, 1),
(763, 767, 1, 'Husna AA', '', '', '', 1349924206, 0),
(764, 768, 1, 'Robaiyah K', '', '', '', 1349924207, 0),
(765, 769, 1, 'Tanti IR', '', '', '', 1349924207, 0),
(766, 770, 1, 'Abdul Fattah AW', '', '', '', 1349927292, 0),
(767, 771, 1, 'Muhammad Razif MY', '', '', '', 1349927292, 0),
(768, 772, 1, 'Primuharsa Putra SHA', '', '', '', 1349927292, 0),
(769, 773, 1, 'Oranye NO', '', '', '', 1349931535, 0),
(770, 774, 1, ' Utharas A', '', '', '', 1349931535, 0),
(771, 775, 1, 'Nora A', '', '', '', 1349931535, 0),
(772, 776, 1, 'Wan Salwina WI', '', '', '', 1349932379, 0),
(773, 777, 1, 'Raynuha M', '', '', '', 1349932380, 0),
(774, 778, 1, 'Idayu BI', '', '', '', 1349932380, 0),
(775, 779, 1, 'Mohd Tahir J', '', '', '', 1349933418, 0),
(776, 780, 1, 'Gendeh BS', '', '', '', 1349933418, 0),
(777, 781, 1, 'Mohd Razif MY', '', '', '', 1349933418, 0),
(778, 782, 1, 'Ruth PRD', '', '', '', 1349945593, 0),
(779, 783, 1, 'Santhna LP', '', '', '', 1349945593, 0),
(780, 784, 1, 'Maraei Awadh BM', '', '', '', 1349946551, 0),
(781, 785, 1, 'Elnasri MA', '', '', '', 1349946551, 0),
(782, 786, 1, 'Tan SP', '', '', '', 1349946551, 0),
(783, 787, 1, 'Noraidah M', '', '', '', 1349946551, 0),
(784, 788, 1, 'Shahrun NS', '', '', '', 1349955548, 0),
(785, 789, 1, 'Hamidah A', '', '', '', 1349958817, 0),
(786, 790, 1, 'Mazlina M', '', '', '', 1349958817, 0),
(787, 791, 1, 'Muhaizan WM', '', '', '', 1349958817, 0),
(788, 792, 1, 'Fuad I', '', '', '', 1349958818, 0),
(789, 793, 1, 'Jamal R', '', '', '', 1349958818, 0),
(790, 794, 1, 'Loke CY', '', '', '', 1349961256, 0),
(791, 795, 1, 'Nur Hidayah MS', '', '', '', 1349961257, 0),
(792, 796, 1, 'Mohd Fadhli MF', '', '', '', 1349961257, 0),
(793, 797, 1, 'Teo SK', '', '', '', 1349961257, 0),
(794, 798, 1, 'Nor Hidayah AG', '', '', '', 1349961257, 0),
(795, 799, 1, 'Alfizah H', '', '', '', 1349962417, 0),
(796, 800, 1, 'Rizal AM', '', '', '', 1349962417, 0),
(797, 801, 1, 'Isa MR', '', '', '', 1349962417, 0),
(798, 802, 1, 'Aminudin A', '', '', '', 1349962417, 0),
(799, 803, 1, 'Jasmi AY', '', '', '', 1349962417, 0),
(800, 804, 1, 'Ramelah M', '', '', '', 1349962417, 0),
(801, 805, 1, 'Fatimah AB', '', '', '', 1349967349, 0),
(802, 806, 1, 'Noor Azah A', '', '', '', 1349967349, 0),
(803, 807, 1, 'Amaramalar SN', '', '', '', 1349967349, 0),
(804, 808, 1, 'Norlaila M', '', '', '', 1349967349, 0),
(805, 809, 1, 'Halimuddin S', '', '', '', 1349967796, 0),
(806, 810, 1, 'Ong TC', '', '', '', 1349968163, 0),
(807, 811, 1, 'Chang KM', '', '', '', 1349968163, 0),
(808, 812, 1, 'Puteri Jamilatul NMB', '', '', '', 1349968163, 0),
(809, 813, 1, 'Poulsaeman v', '', '', '', 1349968539, 0),
(810, 814, 1, 'Suria AA', '', '', '', 1349968539, 0),
(811, 815, 1, 'Faizah AR', '', '', '', 1349969008, 0),
(812, 816, 1, 'Jeevanan J', '', '', '', 1349969008, 0),
(813, 817, 1, 'Hartini Y', '', '', '', 1349969701, 0),
(814, 818, 1, 'Mohamed Kamel AG', '', '', '', 1349969701, 0),
(815, 819, 1, 'Sayyidi Hamzi AR', '', '', '', 1349988585, 0),
(816, 820, 1, 'Norin Rahayu S', '', '', '', 1349988585, 0),
(817, 821, 1, 'Shamsul Azhar S', '', '', '', 1349988585, 0),
(818, 822, 1, 'Syed Mohamed A', '', '', '', 1349988585, 0),
(819, 823, 1, 'Noor Siah AA', '', '', '', 1349989801, 0),
(820, 824, 1, 'Zaidah Z', '', '', '', 1349989801, 0),
(821, 825, 1, 'Teoh KH', '', '', '', 1349989802, 0),
(822, 826, 1, 'Gurbinder JS', '', '', '', 1349989803, 0),
(823, 827, 1, 'Ismail MS', '', '', '', 1349989803, 0),
(824, 828, 1, 'Mazeni A', '', '', '', 1349989803, 0),
(825, 829, 1, 'Zahirrudin Z', '', '', '', 1349990176, 0),
(826, 830, 1, 'Bastion MLC', '', '', '', 1349990873, 0),
(827, 831, 1, 'Siti Aishah S', '', '', '', 1349990873, 0),
(828, 832, 1, 'Barkeh HJ', '', '', '', 1349990873, 0),
(829, 833, 1, 'Roslenda AR', '', '', '', 1350031223, 0),
(830, 834, 1, 'Iqbal FRW', '', '', '', 1350031223, 0),
(831, 835, 1, 'Juriza I', '', '', '', 1350032615, 0),
(832, 836, 1, 'Sharifah Azween SO', '', '', '', 1350032615, 0),
(833, 837, 1, 'Azli I', '', '', '', 1350032615, 0),
(834, 838, 1, 'Mohd Fadly MA', '', '', '', 1350032615, 0),
(835, 839, 1, 'Chia WK', '', '', '', 1350032615, 0),
(836, 840, 1, 'Clarence Ko CH', '', '', '', 1350032615, 0),
(837, 841, 1, 'Julia MI', '', '', '', 1350032615, 0),
(838, 842, 1, 'Khairunisa K', '', '', '', 1350032615, 0),
(839, 843, 1, 'Sharifah Noor Akmal SH', '', '', '', 1350032615, 0),
(840, 844, 1, 'Leong CF', '', '', '', 1350035124, 0),
(841, 845, 1, 'NH Hamidah', '', '', '', 1350035124, 0),
(842, 846, 1, 'Nur Azlina MF', '', '', '', 1350057333, 0),
(843, 847, 1, 'Kamisah Y', '', '', '', 1350057333, 0),
(844, 848, 1, 'Qodriyah MS', '', '', '', 1350057333, 0),
(845, 849, 1, 'Azma AY', '', '', '', 1350057333, 0),
(846, 850, 1, 'Alini M', '', '', '', 1350057333, 0),
(847, 851, 1, 'Avery-Kiejda KA', '', '', '', 1350062293, 0),
(848, 852, 1, 'Wong MW', '', '', '', 1350062293, 0),
(849, 853, 1, 'Scott RJ ', '', '', '', 1350062293, 0),
(850, 854, 1, 'Ivyna Bong PN', '', '', '', 1350213757, 0),
(851, 855, 1, 'Nor Aina E', '', '', '', 1350213757, 0),
(852, 856, 1, 'Noor Hisham A', '', '', '', 1350213757, 0),
(853, 857, 1, 'Liew AKC', '', '', '', 1350240955, 0),
(854, 858, 1, 'Abdul Kadir R', '', '', '', 1350240955, 0),
(855, 859, 1, 'Pua SC', '', '', '', 1350240955, 0),
(856, 860, 1, 'How RA', '', '', '', 1350240955, 0),
(857, 861, 1, 'Lim KH', '', '', '', 1350242149, 0),
(858, 862, 1, 'Sumarni MG', '', '', '', 1350242149, 0),
(859, 863, 1, 'Kee CC', '', '', '', 1350242149, 0),
(860, 864, 1, 'Amal NM', '', '', '', 1350242149, 0),
(861, 865, 1, 'Norhamimah A', '', '', '', 1350242149, 0),
(862, 866, 1, 'Nor Rizan K', '', '', '', 1350264634, 0),
(863, 867, 1, 'Abdul Manaf A', '', '', '', 1350264635, 0),
(864, 868, 1, 'Sabariah AR', '', '', '', 1350264635, 0),
(865, 869, 1, 'Noorjahan Banu MA', '', '', '', 1350264635, 0),
(866, 870, 1, 'Aidifitrina RK', '', '', '', 1350297988, 0),
(867, 871, 1, 'Faizah MZ', '', '', '', 1350342046, 0),
(868, 872, 1, 'Noorakmal A', '', '', '', 1350342046, 0),
(869, 873, 1, 'Syariz IS', '', '', '', 1350342046, 0),
(870, 874, 1, 'Dayang AA', '', '', '', 1350342047, 0),
(874, 878, 1, 'mosmul', '', '', '', 1354942552, 1),
(876, 880, 1, 'zuraini', '', '', '', 1355185860, 1),
(893, 897, 1, 'Farah Sham', '', '', '', 1356069259, 1),
(896, 900, 1, 'Review of Tai Chi Exercise Modulates Physiological and Biochemical Parameters in Pre-Menopausal Women 2', '', '', '', 1358295779, 0),
(897, 901, 85, 'Hanita Othman', '', '', '', 1357705643, 1),
(899, 903, 86, 'Review of Testing-Clinical value of “27 minutes”, fully automated thyroid autoantibody assay in the diagnosis and management of Graves disease.', '', '', '', 1357715008, 0),
(900, 905, 1, 'Clinical value of “27 minutes”, fully automated thyroid autoantibody assay in the diagnosis and management of Graves disease.', '<p>\r\n	The most common cause of hyperthyroidism is Graves disease (GD) which is characterised by the presence of autoantibodies which binds to the TSH receptor (TRAb). Recently, a rapid, fully automated electrochemiluminescent immunoassay Elecsys&acirc;Anti-TSHR for detection of autoantibodies to TSH receptor was made available for routine clinical used. The objective of this study is to evaluate this assay and to determine the sensitivity, specificity and cut-off value. Interassay and total imprecision (CV) were determined at 3.78-7.02 IU/L and 13.5-21.2 IU/L respectively. A total of 124 samples which comprised of 46 GD, seven Hashimoto thyroiditis (HD), 11 non autoimmune nodular goitre (NAG), 2 thyroid cancer (Ca) and 58 normal controls were retrospectively analysed to determine the sensitivity, specificity and cut-off value. Inter-assay CV&rsquo;s were 2.4% at a concentration of 3.90 IU/L (range: 3.78-7.02 IU/l) and 0.8% at 20.80 IU/L (range:13.5-21.2 IU/l) Total imprecision was 3.8% at a concentration of 3.80 IU/L (range:13.5-21.2 IU/l) and 1.0% at 20.8 IU/L (range:13.5-21.2 IU/l). The ROC analysis of patients with GD, other thyroid disorders and normal controls revealed that the highest sensitivity (94%) and specificity (98%) were seen at cut-off value of 1.69 IU/L. Positive predictive value (PPV) and negative predictive value (NPV) was 95% and 94% respectively. At this derived cut-off value of 1.69 IU/L, we found that the sensitivity of TRAb positivity within the group of 29 newly diagnosed GD patients was 94%. Our results demonstrate that this fully automated assay with testing time of 27 minutes has high sensitivity in detecting GD and high specificity for discriminating from other thyroid disease and represent major improvement in the diagnosis and management of patients with thyroid diseases.</p>\r\n', '<p>\r', '', 1374521634, 1),
(901, 906, 64, 'lina', '', '', '', 1358129475, 1),
(902, 907, 88, 'Chia Wai Kit', '', '', '', 1358321895, 1),
(903, 908, 64, '3p25 Aneusomy and Underexpression of PPARγ Protein in Follicular Thyroid Neoplasms: A Report of Three Cases with Review of Literature', '<p>\r\n	Aneusomy is an early genetic event and a characteristic feature of many solid tumors. It is often associated with poor prognosis in cancer patients. The involvement of PAX8-PPAR&gamma; rearrangement in tumorigenesis of follicular thyroid lesions has been widely assessed. However, there were few reports on aneusomy of the PPAR&gamma; gene at the 3p25 locus in follicular thyroid lesions. It remains undetermined whether these abnormalities can be translated into improved diagnosis, classification, or outcome prediction. Herein, we report three cases of follicular thyroid neoplasms [two follicular thyroid carcinomas (FTCs) and one Hurthle cell adenoma (HCA)] with 3p25 aneusomy detected by fluorescence in situ hybridization (FISH). 3p25 trisomy was observed in one FTC and one HCA while 3p25 tetrasomy was observed in one FTC. Furthermore, all three lesions did not show overexpression of PPAR&gamma; protein. Hurthle cell neoplasms (HCN) are distinct clinically and histologically from other follicular thyroid neoplasms (FTN). However, the presence of the aneusomy in HCA and FTC indicates that there could be a biological continuum between the two and chromosomal gains might play an important role in the pathogenesis of these two types of neoplasms. Despite their differences, HCN and FTN may share the same early genetic event in tumor development.</p>\r\n', '<p>\r', '', 1358322577, 1),
(904, 909, 89, 'Review of 3p25 Aneusomy and Underexpression of PPARγ Protein in Follicular Thyroid Neoplasms: A Report of Three Cases with Review of Literature', '', '', '', 1358324611, 0),
(907, 912, 5, 'Testing Submission', '<p>\r\n	testing submission submissions testing testing submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testing testing submission submissions testing testing submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testing testing submission submissions testing testing submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testing testing submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testingtesting submission submissions testing</p>\r\n', '<p>\r', '', 1359195271, 1),
(908, 913, 5, 'Review of Testing Submission', '', '', '', 1359195825, 0),
(909, 914, 64, 'Vol. 7 No. 1 : 2012', '', '', '', 1360035288, 1),
(910, 915, 64, 'JOHAR MJ', '', '', '', 1359620971, 0),
(911, 916, 64, 'BALA KRISHNIAN M', '', '', '', 1359620972, 0),
(912, 917, 64, 'MOHD RADHI A', '', '', '', 1359620972, 0),
(913, 918, 64, 'SAADAH NA', '', '', '', 1359620972, 0),
(914, 919, 64, 'GAN KB', '', '', '', 1359620972, 0),
(915, 920, 64, 'Development of the Pelvic and Lower Limb Immobilization Device: A Universiti Kebangsaan Malaysia (UKM) Innovation Project', '<p>\r\n	Uncontrolled bleeding due to pelvic fractures contributes to trauma-related morbidity and mortality. Three main strategies that have been outlined to combat this condition which include reduction of pelvic volume that lead to tamponade-like effect, arresting haemorrhage through angioembolization of the major vessels, and stabilization of the pelvic bone with external fixation need to be initiated early. A prehospital device that allow these strategies will aid significantly in the management of the patient. At present most devices used to treat pelvic fractures in the pre-hospital setting do have its&rsquo; own advantages but also have some limitations. A characteristic &lsquo;wish-list&rsquo; of a good pelvic and lower limb immobilization device was created and the research team from UKM takes the challenge to design and produce a device that concurs to it. A two phase development project that incorporate anthropometric, biomechanical, cadaveric and radiological study was carried out over a period of seven years. Finally, BRIMTM immobilizer, a new pelvic and lower limb immobilization device that is user friendly, tough, cost effective, radiolucent, light and reusable that answers most of the requirement of a good device was invented.</p>\r\n', '<p>\r', '', 1360035356, 1),
(916, 921, 64, 'HIZLINDA T', '', '', '', 1359945365, 0),
(917, 922, 64, 'SITI NURBAIYAH KE', '', '', '', 1359945365, 0),
(918, 923, 64, 'AZRINA AS', '', '', '', 1359945365, 0),
(919, 924, 64, 'MOHAMAD HAFIZZUDIN MT', '', '', '', 1359945365, 0),
(920, 925, 64, 'CHANG LH', '', '', '', 1359945366, 0),
(921, 926, 64, 'NORALIZA MA', '', '', '', 1359945366, 0),
(923, 928, 64, 'HIZLINDA T, , , , , , ', '', '', '', 1359945539, 0);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(924, 929, 64, 'A Cross-Sectional Study on Patient Satisfaction with Universiti Kebangsaan Malaysia Medical Centre (UKMMC) Primary Care Clinic', '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Patient satisfaction plays a crucial role in ensuring utilization of healthcare services, continuity of care, and compliance towards treatment. Thus, this study aimed at determining the level of patient satisfaction with the services provided by the UKMMC primary care clinic. A descriptive cross-sectional study involving 317 patients attending the clinic from February to March 2011 was carried out. They were selected through systematic random sampling. Using a validated self-administered Patient Satisfaction Questionnaire-46 (PSQ-46) in English and Malay versions, general satisfaction and satisfactions with five different subscales (doctors, nurses, accessibility, facilities and appointment) were assessed. A majority of the patients (93.1%) were generally satisfied with the overall services. Among the five subscales, patients were most satisfied with the doctors (96.5%), but only 35.6% of the patients were satisfied with the facilities. The proportions of patients who were satisfied with other subscales were: 86.1% (appointment), 82.0% (nurses) and 68.1% (accessibility). There was a significant association between each of the subscales and the general satisfaction (p&lt;0.001 for doctors, nurses, accessibility and appointment; p=0.014 for facilities). However, such relationship was not demonstrated between the socio-demographic factors and the general satisfaction (p=0.200 for gender, p=0.108 for age, p=0.894 for race, p=0.146 for educational level, p=0.653 for job status, p=0.867 for type of occupation, and p=0.623 for family income). In conclusion, many patients perceived that clinic facilities were substandard but they still had a high level of general satisfaction. Nevertheless, strategies in improving the facilities are still required as it will contribute to the quality of care and patient satisfaction.</span></p>\r\n', '<p>\r', '', 1360035392, 1),
(925, 930, 64, 'HANITA O', '', '', '', 1359946174, 0),
(926, 931, 64, 'AZURA NR', '', '', '', 1359946174, 0),
(927, 932, 64, 'FAIZAL MMZ', '', '', '', 1359946174, 0),
(928, 933, 64, 'Clinical Value of a Rapid Fully Automated Thyroid Autoantibody Assay in the Diagnosis and Management of Graves Disease', '<p>\r\n	<span style="font-size: 12px; line-height: normal;">The most common cause of hyperthyroidism is Graves disease (GD) which is characterised by the presence of autoantibodies which binds to the TSH receptor (TRAb). Recently, a rapid, fully automated electrochemiluminescent immunoassay ElecsysAnti-TSHR for detection of autoantibodies to TSH receptor was made available for routine clinical use. The objective of this study is to evaluate this assay and to determine the sensitivity, specificity and cut-off value. Interassay and total imprecision (CV) were determined at 3.78-7.02 IU/L and 13.5-21.2 IU/L respectively. A total of 124 samples which comprised of 46 GD, seven Hashimoto thyroiditis (HD), 11 non autoimmune nodular goitre (NAG), 2 thyroid cancers (Ca) and 58 normal controls were retrospectively analysed to determine the sensitivity, specificity and cut-off value. Inter-assay CV&rsquo;s were 2.4% at a concentration of 3.90 IU/L (range: 3.78-7.02 IU/l) and 0.8% at 20.80 IU/L (range:13.5-21.2 IU/l). Total imprecision was 3.8% at a concentration of 3.80 IU/L (range:13.5-21.2 IU/l) and 1.0% at 20.8 IU/L (range:13.5-21.2 IU/l). The ROC analysis of patients with GD, other thyroid disorders and normal controls revealed that the highest sensitivity (94%) and specificity (98%) were seen at cut-off value of 1.69 IU/L. Positive predictive value (PPV) and negative predictive value (NPV) was 95% and 94% respectively. At this derived cut-off value of 1.69 IU/L, we found that the sensitivity of TRAb positivity within the group of 29 newly diagnosed GD patients was 94%. Our results demonstrate that this fully automated assay with testing time of 27 minutes has high sensitivity in detecting GD and high specificity for discriminating other thyroid disease and represent major improvement in the diagnosis and management of patients with thyroid diseases.</span></p>\r\n', '<p>\r', '', 1360035407, 1),
(929, 934, 64, 'MDZIN R', '', '', '', 1359946630, 0),
(930, 935, 64, 'LAU TY', '', '', '', 1359946631, 0),
(931, 936, 64, 'Inverse Correlation between P53 and Bcl-2 Expression in Breast Carcinoma of Malaysian Patients', '<p>\r\n	<span style="font-size: 12px;"><span style="line-height: normal;">The tumour suppressor gene p53 and the proto-oncogene Bcl-2 encode respectively, for a nuclear phosphoprotein and for a mitochondrial membrane protein involved in multiple cellular functions. Both proteins are linked to programmed cell death pathways and provide prognostic information on breast carcinoma. Our aim is to study the expression of p53 and Bcl-2 oncoproteins in breast carcinoma and correlate with patients&rsquo; age, tumour size, disease stage and histological grade. Fifty nine cases of breast carcinomas from Universiti Kebangsaan Malaysia Medical Centre (UKMMC) were studied with the immunohistochemical method. Our results&nbsp;</span><span style="line-height: 1.7em;">showed 45.8% (27 of 59) and 40.7% (24 of 59) of the breast carcinomas were immunopositive for p53 and Bcl-2 respectively. There was significant correlation between Bcl-2 expression with early tumour stage (p=0.01). No significant relationship was seen with other variables. Results also showed an inverse relationship between p53 and Bcl-2 expression (p=0.001). These findings indicate a down regulation of Bcl-2 by p53 in breast carcinogenesis.</span></span></p>\r\n', '<p>\r', '', 1360035421, 1),
(932, 937, 64, 'NURDIYANA MN', '', '', '', 1359947240, 0),
(933, 938, 64, 'HUIK MAY L', '', '', '', 1359947241, 0),
(934, 939, 64, 'ALEX YCS', '', '', '', 1359947241, 0),
(935, 940, 64, 'NOORNABILLAH R', '', '', '', 1359947241, 0),
(936, 941, 64, 'HUD MA', '', '', '', 1359947241, 0),
(937, 942, 64, 'NORAZLIN MI', '', '', '', 1359947241, 0),
(938, 943, 64, 'NURASYIKIN Y', '', '', '', 1359947242, 0),
(939, 944, 64, 'Red Cell Antibody Screening in Pregnancy: A Preliminary Insight?', '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Red cell alloimmunisation is defined as the development of antibodies in response to foreign red cell antigens through transfusion or pregnancy. In pregnant women even without the history of previous blood transfusion, this is possible through previous or current pregnancy with the presence of paternal red cell antigen inherited by the fetus. This study was aimed to determine the prevalence of red cell alloimmunisation among pregnant women without previous history of blood transfusion and the association with number of pregnancy and history of obstetric complications. This was a cross-sectional study in which 150 pregnant women were randomly selected from the antenatal clinic. Ten mls of peripheral blood was obtained for antibody screening using indirect antiglobulin test besides the routine antenatal screening. In this study, the majority (37.3%) of the women were primigravidae. Red cell alloantibodies were detected in two out of 150 (1.3%) patients which were subsequently identified as anti-C and anti-D. However none of the primigravida was alloimmunised. One woman of gravida 2 (2.9%) and gravida 3 (3.6%) each were positive for alloimmunisation. One of them also had a bad obstetric history. This study showed that the prevalence of red cell alloimmunisation among pregnant women was low in this centre. Nevertheless, red cell alloantibody screening test should be made available to reduce possible complications of alloimmunisation in mothers and fetuses.</span></p>\r\n', '<p>\r', '', 1360035445, 1),
(940, 945, 64, 'UMI KALTHUM MN', '', '', '', 1359948393, 0),
(941, 946, 64, 'Dengue Fever Presenting as Bilateral Dengue Maculopathy', '<p>\r\n	Dengue fever is a common pyrexial-viral infection in the Asian region and the incidence is increasing yearly. There are various ocular manifestations of dengue fever such as maculopathy, retinal hemorrhages, retinal lesions and vasculitis. Reduction in visual acuity and other visual disturbances such as metamorphopsia is the most common presentation of dengue-associated maculopathy. We report an interesting case of a young lady with bilateral maculopathy secondary to dengue fever who was managed conservatively with dramatic visual improvements. &nbsp;This case highlights the possibility of bilateral and simultaneous ocular involvements in patients infected with dengue fever. Both ophthalmologists and physicians need to be aware of ocular complications following dengue fever and advice patients regarding the symptoms.&nbsp;</p>\r\n', '<p>\r', '', 1360035473, 1),
(942, 947, 64, 'REENA RAHAYU MZ', '', '', '', 1359963406, 0),
(943, 948, 64, 'ASMIATI A', '', '', '', 1359963406, 0),
(944, 949, 64, 'RAFIE MK', '', '', '', 1359963406, 0),
(945, 950, 64, '3p25 Aneusomy in Follicular Thyroid Neoplasms: A Report of Three Cases with Review of Literature', '<p>\r\n	<span style="font-size: 12px; line-height: normal;">Aneusomy is an early genetic event and a characteristic feature of many solid tumors. It is often associated with poor prognosis in cancer patients. The involvement of PAX8-PPAR&gamma; rearrangement in tumorigenesis of follicular thyroid lesions has been widely assessed. However, there were few reports on aneusomy of the PPAR&gamma; gene at the 3p25 locus in follicular thyroid lesions. It remains undetermined whether these abnormalities can be translated into improved diagnosis, classification, or outcome prediction. Herein, we report three cases of follicular thyroid neoplasms [two follicular thyroid carcinomas (FTCs) and one Hurthle cell adenoma (HCA)] with 3p25 aneusomy detected by fluorescence in situ hybridization (FISH). 3p25 trisomy was observed in one FTC and one HCA while 3p25 tetrasomy was observed in one FTC. Furthermore, all three lesions did not show overexpression of PPAR&gamma; protein. Hurthle cell neoplasms (HCN) are distinct clinically and histologically from other follicular thyroid neoplasms (FTN). However, the presence of the aneusomy in HCA and FTC indicates that there could be a biological continuum between the two and chromosomal gains might play an important role in the pathogenesis of these two types of neoplasms. Despite their differences, HCN and FTN may share the same early genetic event in tumour development.</span></p>\r\n', '<p>\r', '', 1360222489, 1),
(946, 951, 64, 'testing-lina', '<p>\r\n	tes tingte stin gtestin gtestingtes tingte st ingte stingte stingtesti ngte stingte stin gtesti ngte stingtes tingte sting testing testingte st ingte stingt estin gte stingte stingte stingte sting test ingtestingtestingtestingtestingtestingtestingtestingte stin gte sting tes in gtes tingte s t ngtestin t estingtestingt estingtestingtestingtestingtestin gtestingte stingtestingtesting t estingtestin gt estin gtestin g testin gt es tingtestingt esti ng testingt estingtestingtest ingtestingtesting testingtestingtestingtes tingtestingtesting testingtesting testingte stingt estingtestin gtestingtest ingtestingt esting testi ngtestin testi ngtestin gtestin gtestingte stingtestingtes tingtest ingtestingtesting testingtes ingt estingte stin gtestingte st ingtest ingt estingte stingtestingtestingtestingtestingtestin gtes tingtes tingtesting testingtestingte stingtestingtestingtest ingtestingtestingtestingtesti ngtestingtestingtestin testingt estingtest ing</p>\r\n', '<p>\r', '', 1376900382, 1),
(947, 952, 86, 'Review of testing-lina', '', '', '', 1360117738, 0),
(948, 953, 86, 'Review of Clinical value of “27 minutes”, fully automated thyroid autoantibody assay in the diagnosis and management of Graves disease.', '', '', '', 1360117788, 0),
(949, 954, 64, 'Comparative Antioxidant and Anti-inflammatory Activity of Different Extracts of Centella asiatica (L.) Urban and Its Active Compounds, Asiaticoside and Madecassoside. ', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	The potential of <em>Centella asiatica</em> (CA) as an antioxidant and anti-inflammatory agents has &nbsp;been well described. However the extraction method which gives the best yield is debatable. In this study, we evaluated three different methods of extractions and compared the extracts in terms of antioxidant, anti-inflammatory activities as well as the contents of its bioactive compounds, asiaticoside and madecassoside. <em>Centella asiatica</em> was extracted using ethanol, methanol and aqueous extraction methods. The extracts were then measured for their phenolic contents using Folin-Ciocalteu reagent. Asiaticoside and madecassoside were determined using HPLC. Antioxidant &nbsp;activity was measured using the 2,2-diphenyl-1-picrylhydrazyl (DHPP) and ferric reducing antioxidant power (FRAP) assays. Anti-inflammatory activities were determined by the ability of the extracts to inhibit the inflammatory pathway enzyme, COX-1 and COX-2 as well as their ability to protect fibroblasts against 12-<em>O</em>-tetradecanoylphorbol-13-acetate (TPA) -induced production of prostaglandin E<sub>2</sub> (PGE<sub>2</sub>). Results showed that the level of phenolic constituents, asiaticoside and madecassoside were highest in the ethanol, followed by methanol and then aqueous extracts (17.76 g/100g, 15.52 g/100g, 13.16 g/100g for phenolics, 42.86 mg/g, 36.37 mg/g, 2.82 mg/g for asiaticoside and 18.66 mg/g, 15.87 mg/g, 3.75 mg/g for madecassoside respectively. All extracts showed considerable antioxidant activity compared to the positive controls. The extracts, asiaticosside and madecassoside &nbsp;&nbsp;inhibited both COX-1 and COX-2 and suppressed the TPA-induced production of PGE<sub>2</sub>. The ethanol and methanol extracts were stronger COX inhibitors and more potent suppressor of PGE<sub>2 </sub>formation than aqueous extract. Thus although the aqueous extract showed higher antioxidant potential, &nbsp;in terms of anti-inflammatory activities, the hydrophobic solvents, ethanol and methanol, proved to be the better extraction method for <em>Centella asiatica</em>.</p>\r\n', '<p>\r\n	&nbsp;</p>', '', 1360915618, 1),
(950, 955, 1, 'Guide for Author - EJournal system', '\r\n', '\r\n', '', 1361148033, 1),
(951, 956, 64, 'Biliary Ascariasis from a rural setting: A case study', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Ascariasis is a common helminthic infestation in Malaysia, particularly in rural settings. Ascariasis lumbricoides normally lives in the upper small bowel without causing symptoms. Complications arise when these worms migrate into the bile duct (biliary ascariasis). A case of biliary ascariasis presenting as biliary colic is described. Patient presented with 2 days duration of right upper quandrant pain at the district hospital. Initial investigations were suggestive of acute cholecystits and patient was treated with empirical antibiotics. However due to worsening symptoms, she was transferred to nearest tertiary setting. The diagnosis of helminith biliary infestation was established using both ultrasound of the bepato-biliary system (HBS), CT-Scan Abdomen and endoscopic retrograde cholangio-pancreatography (ERCP). Endoscopic removal of the worm led to rapid resolution of symptoms and patient was discharge home well.&nbsp;</p>\r\n', '<p>\r\n	&nbsp;</p>', '', 1361325056, 1),
(952, 957, 97, 'Review of Biliary Ascariasis from a rural setting: A case study', '', '', '', 1361369167, 0),
(953, 958, 1, 'Guide for Reviewer - EJournal system', '', '', '', 1361855710, 1),
(954, 959, 104, 'Che Hassan Hamat Hamdi', '', '', '', 1363838712, 1),
(955, 960, 104, 'Osama Ali', '', '', '', 1363838738, 1),
(956, 961, 64, 'Impact on Knowledge in Dengue Management among Medical and Paramedical personnel following attendance to Educational Lectures', '<p>\r\n	This study was carried out to assess the effectiveness of dengue talks to raise and improve knowledge of health care providers. This is a descriptive cross sectional study consisting of a pre and post test evaluation. Comparison of data was done with the McNemar test and independent T test. There was significant improvement in all knowledge sections among the overall health providers group. Pre test scores mean was higher among the doctors compared to paramedics. However, post test scores showed the margin of differences narrowed, where the difference of scores in paramedics is larger than the doctors. There was a statistical improvement of all knowledge scores after the dengue talks. The improvement was more pronounced in the paramedic group compared to the doctors.</p>\r\n', '<p>\r', '', 1364265388, 1),
(957, 962, 101, 'Nik Azlan Nik Muhamad', '', '', '', 1363852788, 1),
(958, 963, 101, 'azizol mahmud', '', '', '', 1363853319, 1),
(959, 964, 64, 'Photoprotective Measures of Selected Sunscreens and Its Antioxidants Adjuvantion Effects in Malaysian Climate', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	Malaysia skincare industry is growing fast seeing vast number of new sunscreens had been produced annually. Inadequacy of skincare regulations and lacking in its enforcement, amongst others has resulted in the overrating of these sunscreens&rsquo; quality. The objectives of this study were to evaluate the efficacy and safety of the local and international sunscreens available in Malaysian market and to determine the augmentation effects of adding concentrated antioxidants into them. Three local and three international manufactured sunscreens were tested using a modified COLIPA method for the in vitro determination of Ultraviolet A protection factor (UVAPF) 2009. The cream samples were spread onto roughened polymethylmethacrylate (PMMA) plates where the absorbance profiles of the samples before and after two hours of sunlight irradiation were measured using a spectrophotometer for the determination of in vitro Sun Protection Factor (SPF), UVAPF and photostability. The same procedure was repeated on&nbsp; the sunscreens with addition of commercially available concentrated vitamin C and E creams. International branded sunscreens were found to have more accurate SPF labels (p=0.009) while local branded sunscreens were found to be more photostable (p=0.003). However, both&nbsp; sunscreens did not provide adequate UVAPF (p=0.471). Addition of vitamin C enhanced the SPF&nbsp; values (p= 0.04) of both groups of sunscreens while vitamin E enhanced their photostability (p=0.000). Interestingly, adding a combination of vitamins C and E&nbsp; into the sunscreens did not&nbsp; improve the SPF and UVAPF values as compared to the use of a single vitamin. More importantly, the combination of vitamins decreased the photostability (p=0.002) of sunscreens as compared to addition of vitamin E alone. In conclusion, photoprotection is best achieved when sunscreens are used together with creams containing either vitamin C or E alone.</p>\r\n', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	&nbsp;</p>', '', 1363936834, 1),
(960, 965, 97, 'Review of Photoprotective Measures of Selected Sunscreens and Its Antioxidants Adjuvantion Effects in Malaysian Climate', '', '', '', 1363948661, 0),
(961, 966, 64, 'Management of surge in critically ill patients in a tertiary teaching hospital', '<p>\r\n	Emergency department overcrowding has been a longstanding problem. Hospital beds closure or access block to ward admission is the single most important cause of emergency department overcrowding.1 This could be compounded further in events of a patient surges e.g. disaster. A surge response entails even greater responses including implementing affirmative measurement in order to mitigate the issue in tackling the situation</p>\r\n', '<p>\r\n	Emergency department overcrowding has been a longstanding problem. Hospital beds closure or access block to ward admission is the single most important cause of emergency department overcrowding.1 This could be compounded further in events of a patient surges e.g. disaster. A surge response entails even greater responses including implementing affirmative measurement in order to mitigate the issue in tackling the situation</p>\r\n', '', 1373507956, 1),
(962, 967, 108, 'Review of Photoprotective Measures of Selected Sunscreens and Its Antioxidants Adjuvantion Effects in Malaysian Climate', '', '', '', 1364732636, 0),
(963, 968, 109, 'Review of Impact on Knowledge in Dengue Management among Medical and Paramedical personnel following attendance to Educational Lectures', '', '', '', 1365048330, 0),
(964, 969, 109, 'Review of Impact on Knowledge in Dengue Management among Medical and Paramedical personnel following attendance to Educational Lectures', '', '', '', 1365048480, 0),
(965, 970, 4, 'Review of Tai Chi Exercise Modulates Physiological and Biochemical Parameters in Pre-Menopausal Women 2', '', '', '', 1375409550, 0),
(966, 971, 101, 'RUTH SABRINA BINTI SAFFERI', '', '', '', 1365126594, 1),
(967, 972, 101, 'adi osman', '', '', '', 1365130185, 1),
(968, 973, 64, 'ANTERIOR PENETRATING CHEST INJURY COMPLICATED WITH DELAYED LEFT VENTRICULAR THROMBUS EMBOLIZATION', '<p>\r\n	Urban cities in the world had the synonym of having a lot of penetrating chest injuries either from road traffic accidents or from stabbed wounds due to homicidal or suicidal attempts. When this group of patients came to our emergency department resuscitation room, the adrenaline rushed to the maximum in order for us to save the patients&rsquo; life. We are presenting a case report of a patient who presented to the Emergency Department Hospital Raja Permaisuri Bainun, Ipoh, Perak, with complicated anterior penetrating chest injury who had a delayed complication of left ventricular thrombus embolization.</p>\r\n', '<p>\r', '', 1373506752, 1),
(969, 974, 101, 'Haliza Hasan', '', '', '', 1365580739, 1),
(970, 975, 101, 'haliza bte hasan', '', '', '', 1365585213, 1),
(971, 976, 64, 'Haemodialysis Affects Corneal Thickness but Not Intraocular Pressure Measurements', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Objective</strong></p>\r\n<p>\r\n	To<em>study the effect of HD on CCT and IOP in patients undergoing HD </em></p>\r\n<p>\r\n	<strong>Method </strong></p>\r\n<p>\r\n	A hospital-based, prospective observational study was conducted <em>at the Haemodialysis Centre and Ophthalmology Clinic of the UKM Medical Centre (UKMMC), Kuala Lumpur, Malaysia. Fifty </em>eyes of 50 patients undergoing haemodialysis who fulfil the criteria were enrolled in the study using universal sampling method. Ocular examinationwas performed before haemodialysis. Pre- and post-haemodialysis IOP and CCT measurements of between 30 to 60 minutes before and after HD and total body volume measurements were performed for each patient. The recorded IOP measurements were then adjusted for CCT using this formula:Adjusted&nbsp; IOP (aIOP) = measured&nbsp; IOP -(CCT-454)/50 &times; 2.5 mmHg. Out of 50 eyes in total, 45 of right eyes were used for statistical analysis. In 5 cases, the right eyes did not meet the inclusion criteria so the left eyes were used for measurements The data were analysed using the SPSS version 19.0. Paired t-test was used to compare the IOP, CCT and body weight measurements before and after HD. Pearson correlation was used to investigate possible correlation between changes in IOP, CCT and total body volume after HD. A level of p &lt; 0.05 was considered as statistically significant.</p>\r\n<p>\r\n	<strong>Results</strong></p>\r\n<p>\r\n	There were no statistically significant differences in IOP before and after.(p = 0.798). Mean CCT decreased significantly after HD, from 537.0 &plusmn; 38.9 &micro;m to 532.5 &plusmn; 36.2 &micro;m (p = 0.004). No correlation was found between change in CCT and total body volume loss (Pearson correlation test, r = 0.237, p = 0.098), as well as between the change in CCT and change in IOP (p = 0.724).</p>\r\n<p>\r\n	<strong>Conclusion</strong></p>\r\n<p>\r\n	HD causes significant changes in CCT which should be accounted for when measuring the IOP at least 60 minutes after HD, especially in patients being followed for the diagnosis of glaucoma.However, HD does not result in IOP change.</p>\r\n', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<strong>Objective</strong></p>\r\n<p>\r\n	To<em>study the effect of HD on CCT and IOP in patients undergoing HD </em></p>\r\n<p>\r\n	<strong>Method </strong></p>', '', 1366160127, 1),
(972, 977, 64, 'THE PREVALENCE OF INJURIES AMONG PARTICIPANTS DURING THE 16TH NATIONAL SILAT CHAMPIONSHIPS 2012.', '<p>\r\n	Silat is one of several martial art practiced by Indonesian Archipelago and Malay Peninsula of Southeast Asia. The 16th National Silat Championships 2012 is a national level sporting event which was held in an every year interval. Athletes, representing 12 states of Malaysia including army, compete in the event which was held in Kuala Lumpur Badminton Stadium, Cheras from 24-29th of April 2012. The purpose of this study was to provide a descriptive epidemiology about frequency of injuries, characteristics, and type of injuries incurred during the 16th National Silat Championships 2012 competitions. This was a retrospective study by using a Pre Hospital Care Emergency Medicine PPUKM Form. All injuries sustained during the competition days were documented. The total number of participants involved in this tournament was 158 (114 males and 44 females). 51.6% participants sustained various injuries during the competition. The common sites of injuries involved the upper and lower extremities. The injuries sustained consist of swelling, fracture, dislocation, muscle sprain, strain and others. Most of the injuries were minor and did not require hospitalization. This study identified that soft tissue injuries due to blunt trauma were common among the silat participants. However, severe injuries were uncommon. Strict observation of the tournament regulations, vigorous training and protective measures or equipments may contribute to the absence of severe or serious injury.</p>\r\n', '<p>\r', '', 1373445075, 1),
(973, 978, 130, 'Yong Kang Cheah', '', '', '', 1371520468, 1),
(974, 979, 64, 'The Socio-demographic Determinants of Practicing a Healthy Lifestyle: A Case Study in Penang, Malaysia', '<p>\r\n	The primary objective of the present study is to investigate the impact of socio-demographic factors on individuals&rsquo; decision to practice a healthy lifestyle in Penang (Malaysia). A cross-sectional primary survey data with 398 respondents is used for analysis. The present study applies a logistic regression model to examine the factors affecting individuals&rsquo; odds of practicing a healthy lifestyle. The results suggest that gender, ethnicity, marital status, income, education and history of serious family illnesses are significantly associated with individuals&rsquo; propensity to practice a healthy lifestyle. Specifically, individuals who are married and lower educated are more likely to practice a healthy lifestyle, while males, Chinese, higher income earners and individuals who have serious family illnesses are less likely to practice a healthy lifestyle. Based on the findings of the present study, numerous population-based intervention measures are suggested.</p>\r\n', '<p>\r', '', 1373509026, 1),
(975, 980, 64, 'HONEY: FOOD OR MEDICINE?', '<p>\r\n	Honey is a natural substance produced by honeybees, <em>Apis mellifera</em>, from the nectar of blossomed flowers or exudates of trees and plants producing nectar honeys or honeydews, respectively. It is a supersaturated solution of sugars, enriched with proteins, minerals, vitamins, organic acids and polyphenols. Honey possesses numerous nutritional, healing and prophylactic properties attributed by the rich components found in honey. Some of the &nbsp;health beneficial properties include wound healing, antimicrobial, antioxidant and anti-inflammatory potential. This review relates the nutritional composition, antioxidant and therapeutical effects of honey with emphasis on Malaysian honeys.</p>\r\n', '<p>\r', '', 1373440649, 1),
(976, 981, 64, 'Bilateral retinal occlusive vasculitis: infective or immunogenic TB?', '<p>\r\n	A 17- year- old male student of Indonesian parentage presented with two weeks history of progressive painless bilateral visual deterioration. There was no contact with tuberculosis (TB)- infected patients and parents claimed that all immunization including BCG was completed. However, BCG scar was not apparent. Visual acuity was 6/36 and 6/60 in the right and left eyes respectively. The anterior and vitreous chambers were quiet. Funduscopic examination revealed retinal vasculitis with perivascular exudates, branch vein occlusion, neovascularization and macula oedema. Fluorescein angiography study confirmed large areas of capillary non- perfusion and leaking new vessels. Mantoux test was positive and full regime of anti- TB therapy was instituted. HIV screening was negative. Three days later, immunosuppressive dose of oral steroid was started. Both eyes received intensive laser photocoagulation.interestingly, there was no development of vitritis throughout.&nbsp;</p>\r\n', '<p>\r', '', 1373441982, 1),
(977, 982, 64, 'Masquerade syndrome: an ocular involvement of lung cancer', '<p>\r\n	We report a case of a 37 year- old lelaki smoker presented with painless and sudden onset of loss of upper field vision of the right eye, associated with one month history of dry cough which was unresponsive to antibiotic and subsequent anti- tuberculosis therapy. Visual acuity at presentation was counting finger. There was bullous retinal detachment on examination. Radiological imaging revealed multiple lung opacities, involving the brain and the right orbit. Bronchoscopy and biopsy confirmed the diagnosis of adenocarcinoma of the lung. This case illustrates masquerade syndrome of which a lung carcinoma harbours behind a seemingly innocent retinal detachment. It also highlights the importance of entertaining lung carcinoma as a differential diagnosis in suspected tuberculosis among heavy smokers.</p>\r\n', '<p>\r', '', 1373444090, 1),
(978, 983, 4, 'Review of Clinical value of “27 minutes”, fully automated thyroid autoantibody assay in the diagnosis and management of Graves disease.', '', '', '', 1374517356, 0),
(979, 984, 64, 'test', '<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	test</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	test</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	test</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	test</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	test</p>\r\n', '<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>\r\n<p>\r\n	testing testing</p>', '', 1377160053, 1),
(980, 985, 86, 'Review of test', '', '', '', 1377159133, 0),
(981, 986, 150, 'Review of The Socio-demographic Determinants of Practicing a Healthy Lifestyle: A Case Study in Penang, Malaysia', '', '', '', 1378165918, 0),
(982, 987, 97, 'Review of THE PREVALENCE OF INJURIES AMONG PARTICIPANTS DURING THE 16TH NATIONAL SILAT CHAMPIONSHIPS 2012.', '', '', '', 1378199268, 0),
(983, 988, 151, 'Review of THE PREVALENCE OF INJURIES AMONG PARTICIPANTS DURING THE 16TH NATIONAL SILAT CHAMPIONSHIPS 2012.', '', '', '', 1382886968, 0),
(984, 989, 172, 'Shahrul Azmin Md. Rani', '', '', '', 1382948419, 1),
(985, 990, 172, 'Azmin,S', '', '', '', 1382948618, 1),
(968, 992, 101, 'ANTERIOR PENETRATING CHEST INJURY WITH LEFT VENTRICULAR THROMBUS', 'Urban cities are synonym with a high incidence of penetrating chest injuries either from accidents or interpersonal violence. The outcome of penetrating chest wound can vary from immediate death to a prolonged morbidity. We are reporting a case of a 39 year gentleman who presented to Emergency Department Hospital Raja Permaisuri Bainun, Ipoh, Perak after being stabbed to the chest.  His anterior penetrating chest wound was located at the 5th intercostals space medial to the midclavicular line. The stab wound penetrated the myocardium, causing  minimal myocardial rupture. He also suffered from left haemothorax and hemopericardium. The haemothorax was drained with insertion of 32 French chest tube. The patient was admitted under the cardiothoracic team and discharged 5 days later without surgical intervention. He presented again to the Emergency Department with complains of shortness of breath and pleuritic pain. A left ventricular thrombus was detected via echocardiography. Unfortunately he took his own discharge. Five days later he came again to Emergency Department with sporadic of loss of vision. The mural thrombus dislodged and embolized to the retinal artery  causing amaurosis fugax. The patient was treated with aspirin 150mg and his symptoms subsequently resolved.', 'Urban cities are synonym with a high incidence of penetrating chest injuries either from accidents or interpersonal violence. The outcome of penetrating chest wound can vary from immediate death to a prolonged morbidity. We are reporting a case of a 39 year gentleman who presented to Emergency Department Hospital Raja Permaisuri Bainun, Ipoh, Perak after being stabbed to the chest.  His anterior penetrating chest wound was located at the 5th intercostals space medial to the midclavicular line. The stab wound penetrated the myocardium, causing  minimal myocardial rupture.', 'English corrected. Title changed as advised. Searched for similar case but could not find any', 1383031843, 1),
(987, 993, 64, 'Prospective Study Of Functional Recovery Of Stroke Patients At 3 Months Post Admission: Outcomes And Implications For Post Stroke Care Provision.', '<p>\r\n	BACKGROUND Functional recovery in the immediate post stroke period predicts the long-term prognosis of post stroke patients. Despite the advancement in stroke rehabilitation in improving survivor&rsquo;s physical function, there are other factors that may influence functional recovery. We aim to assess the functional recovery of ischaemic stroke patients attending a tertiary hospital and its associated factors in order to make recommendations for post stroke care after hospital. METHODOLOGY A 3-months prospective observational study looking into the functional recovery using the Modified Barthel&rsquo;s Index (MBI) and depression (Patient Heath Questionnaire-9 ) score of post stroke patients. RESULTS There were 46 Ischemic stroke patients that were recruited into the study. At 3 months only 37 patients were able to be analysed. Mean age was 67.2 (SD 11) years with Malays the majority group (50%) followed by Chinese (41.3%) and Indians(8.7%). Total death was 7 (15.2%) deaths. Hypertension was the highest risk factors (89.1%) followed by dyslipidaemia (65.2%) and diabetes (63.0%). The mean MBI different was 45.2 (SD 27.0) with median MBI 17.0(IQR 33.0) at baseline compared to 85.0(IQR 42.0) at 3 months (p &lt; 0.001, CI 35.98,55.45). Prevalence of depression (PHQ-9&ge;10) was 21.6%. Lower functional recovery was found among depressed patients (p &lt;0.026) and among patients with recurrent stroke (p&lt; 0.035). CONCLUSION Significant functional recovery can be seen as early as 3-months post stroke. Factors such as recurrent stroke and depression may affect functional recovery post stroke; hence should be routinely screened.</p>\r\n', '<p>\r', '', 1385430228, 1),
(988, 994, 64, 'Vol. 7 No. 2 : 2012', '', '', '', 1385518988, 1),
(989, 995, 64, 'NURLAILY A', '', '', '', 1385436515, 0),
(990, 996, 64, 'NOOR BAITEE AR', '', '', '', 1385436515, 0),
(991, 997, 64, 'Comparative Antioxidant and Anti-inflammatory Activity of Different Extracts of Centella asiatica (L.) Urban and Its Active Compounds, Asiaticoside and Madecassoside', '<div>\r\n	The potential of Centella asiatica (CA) as an antioxidant and anti-inflammatory agent has been well described. However the extraction method which gives the best yield is debatable. In this study, we evaluated three different methods of extractions and compared the extracts in terms of antioxidant, anti-inflammatory activities as well as the contents of its bioactive compounds, asiaticoside and madecassoside. Centella asiatica was extracted using ethanol, methanol and aqueous extraction methods. The extracts were then measured for their phenolic contents using Folin-Ciocalteu reagent. Asiaticoside and madecassoside were determined using HPLC. Antioxidant activity was measured using the 2,2-diphenyl-1-picrylhydrazyl (DHPP) and ferric reducing antioxidant power (FRAP) assays. Anti-inflammatory activities were determined by the ability of the extracts to inhibit the inflammatory pathway enzyme, COX-1 and COX-2 as well as their ability to protect fibroblasts against 12-O-tetradecanoylphorbol-13-acetate (TPA) -induced production of prostaglandin E2 (PGE2). Results showed that the level of phenolic constituents, asiaticoside and madecassoside were highest in the ethanol, followed by methanol and then aqueous extracts (17.76 g/100g, 15.52 g/100g, 13.16 g/100g for phenolics, 42.86 mg/g, 36.37 mg/g, 2.82 mg/g for asiaticoside and 18.66 mg/g, 15.87 mg/g, 3.75 mg/g for madecassoside respectively. All extracts showed considerable antioxidant activity compared to the positive controls. The extracts, asiaticosside and madecassoside inhibited both COX-1 and COX-2 and suppressed the TPA-induced production of PGE2. The ethanol and methanol extracts were stronger COX inhibitors and more potent suppressor of PGE2 formation than aqueous extract. Thus although the aqueous extract showed higher antioxidant potential, in terms of anti-inflammatory activities, the hydrophobic solvents, ethanol and methanol, proved to be the better extraction method for Centella asiatica.</div>\r\n', '<div>\r', '', 1385452475, 1),
(992, 998, 64, 'RADHIYAH R', '', '', '', 1385436818, 0),
(993, 999, 64, 'RAHMAN J', '', '', '', 1385436818, 0),
(994, 1000, 64, 'Parenting Stress in Childhood Leukaemia', '<div>\r\n	Parenting stress is the stress level experienced within the role of a parent (Hoekstra-Weebers et al. 1998). The source of stressors is variable and dependent on the phase of disease and chemotherapy (Sawyer et al. 2000). Failure to cope with these stressors may in turn affect the child&rsquo;s emotional and social adjustment towards the diagnosis of cancer in addition to poor medical treatment adherence behaviour (Sawyer et al. 1993). The objectives of this study are to determine the level of parenting stress, the risk factors contributing to high parenting stress, and the coping mechanisms used to handle the stress. This single centred, cross-sectional study was done amongst 117 parents at the Paediatric Haematology and Oncology Unit, Universiti Kebangsaan Malaysia Medical Centre (UKMMC) over two years duration. Self-administered questionnaires comprising the Parenting Stress Index/Short Form (PSI/SF) and Coping Inventory for Stressful Situation (CISS) were distributed to parents of children who were 12 years old and below. The mean total parenting stress score amongst parents of children diagnosed with acute leukaemia was 91.5&plusmn;21.1(95% CI). A total of 27.3% of parents experienced a high total parenting stress score (defined as total PSI score &ge; 75th centile, ie &ge; 103). Task-oriented coping mechanism was used by the majority of parents. Emotion-oriented coping mechanism was the only identifiable risk factor for high parenting stress score following multiple logistic regression analysis. A parent who used emotion-oriented coping mechanism was 7.1 times (95% Confidence Interval 1.2 to 41.4) more likely to have a high parenting stress score compared to a parent who used other coping mechanisms. By identifying these at-risk parents, appropriate counselling and psychological support may be offered early to alleviate the stress as well as assist in the coping and adjustment mechanisms of these parents.</div>\r\n', '<div>\r', '', 1385453499, 1),
(995, 1001, 64, 'KHAMSIAH N', '', '', '', 1385437646, 0),
(996, 1002, 64, 'LAI NS', '', '', '', 1385437646, 0),
(997, 1003, 64, 'NURFARHANIM M', '', '', '', 1385437647, 0),
(998, 1004, 64, 'NOR AIMI AR', '', '', '', 1385437647, 0),
(999, 1005, 64, 'MOHD SYAZWAN CS', '', '', '', 1385437647, 0),
(1000, 1006, 64, 'Photoprotective Measures of Selected Sunscreens and Their Antioxidant Adjuvantion Effects in the Malaysian Climate', '<p>\r\n	The Malaysian skincare industry is growing rapidly with a vast number of new sunscreens being produced annually. Inadequate skincare regulations and lack of enforcement have resulted in the overrating of sunscreens&rsquo; quality. The objectives of this study were to evaluate the efficacy and safety of the local and international sunscreens and to determine the effects of adding concentrated antioxidants into them. Three local and three internationally manufactured sunscreens were tested for the in vitro Ultraviolet A protection factor (UVAPF), Sun Protection Factor (SPF) and photostability. The creams were spread onto roughened polymethylmethacrylate plates where the absorbance was measured using a spectrophotometer before and after two hours of sunlight irradiation. The procedure was repeated combining available concentrated vitamin C and E creams. International sunscreens were found to have more accurate SPF labels (p=0.009) while local sunscreens were found to be more photostable (p=0.003). However, both sunscreens had inadequate UVAPF (p=0.471). Vitamin C enhanced the SPF values (p= 0.04) of both groups of sunscreens while vitamin E enhanced their photostability (p=0.000). Interestingly, combining vitamins C and E rich creams with the sunscreens had no effect on the SPF and UVAPF values as compared to the use of a single vitamin. More importantly, the combination of vitamins decreased the photostability (p=0.002) of sunscreens as compared to the addition of vitamin E alone. In conclusion, photoprotection is best achieved when sunscreens are used together with creams containing either vitamin C or E alone.</p>\r\n', '<p>\r', '', 1385453979, 1),
(1001, 1007, 64, 'NORFARIZAL A', '', '', '', 1385438332, 0),
(1002, 1008, 64, 'RONA ASNIDA N', '', '', '', 1385438332, 0),
(1003, 1009, 64, 'AYESHA MZ', '', '', '', 1385438332, 0),
(1004, 1010, 64, 'JEMAIMA CH', '', '', '', 1385438333, 0),
(1005, 1011, 64, 'Bilateral Retinal Vasculitis: A Presumed Case of Ocular TB without Inflammation', '<div>\r\n	A 17-year-old male student of Indonesian parentage presented with two weeks history of progressive painless bilateral visual deterioration. There was no contact with tuberculosis (TB)-infected patients and parents claimed that all immunization including BCG was completed. However, BCG scar was not apparent. Visual acuity was 6/36 and 6/60 in the right and left eyes respectively. The anterior and vitreous chambers were quiet. Funduscopic examination revealed retinal vasculitis with perivascular exudates, branch vein occlusion, neovascularization and macular oedema. Fluorescein angiography confirmed large areas of capillary non-perfusion and leaking new vessels. Mantoux test was positive and full regime anti-TB therapy was instituted. HIV screening was negative. Three days later, an immunosuppressive dose of oral steroid was started. Both eyes received intensive laser photocoagulation.Interestingly, there was no development of vitritis throughout.</div>\r\n', '<div>\r', '', 1385454173, 1),
(1006, 1012, 64, 'SAI SANJEEV M', '', '', '', 1385439655, 0),
(1007, 1013, 64, ' LUM LC', '', '', '', 1385439656, 0);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(1008, 1014, 64, 'Biliary Ascariasis From a Rural Setting: A Case Study', '<div>\r\n	Ascariasis is a common helminthic infestation in Malaysia, particularly in rural settings. Ascariasis lumbricoides normally lives in the upper small bowel without causing symptoms. Complications arise when these worms migrate into the bile duct (biliary ascariasis). A case of biliary ascariasis presenting as biliary colic is described. Patient presented with 2 days duration of right upper quandrant pain at the district hospital. Initial investigations were suggestive of acute cholecystitis and patient was treated with empirical antibiotics. However, due to worsening symptoms, she was transferred to the nearest tertiary setting. The diagnosis of helminthic biliary infestation was established using both ultrasound of the hepato-biliary system (HBS), CT-Scan abdomen and endoscopic retrograde cholangio-pancreatography (ERCP). Endoscopic removal of the worm led to rapid resolution of symptoms and patient was discharged home well.</div>\r\n', '<div>\r', '', 1385454257, 1),
(1009, 1015, 64, 'NATASHA MN,, , , , ', '', '', '', 1385440060, 0),
(1010, 1016, 64, ' KHOO HW', '', '', '', 1385440060, 0),
(1011, 1017, 64, 'SULAIMAN AS', '', '', '', 1385440060, 0),
(1012, 1018, 64, 'MD DALI AZH', '', '', '', 1385440060, 0),
(1013, 1019, 64, 'JAMIL MA', '', '', '', 1385440060, 0),
(1014, 1020, 64, 'Ectopic Pregnancy Following Levonorgesterol-Only Emergency Contraception: The First Malaysian Case Report', '<div>\r\n	Levonorgestrel (LNG) is a well-known safe and efficacious emergency contraception (EC). However, ectopic pregnancy following the failure of LNG-only EC has been reported. The exact incidence of ectopic pregnancy has been hindered by lack of data due to the fact that LNG-only EC is accessible at pharmacies without a prescription. We describe a case of ectopic pregnancy in an 18 year-old single woman who took LNG-only EC within 48 hours of unprotected sexual intercourse. She presented to the emergency department at 8 weeks period of amenorrhoea with an acute abdomen and hypovolaemic shock. Laparotomy confirmed a ruptured right tubal pregnancy and salpingectomy was performed. The patient was discharged well after 2 days. We aim to highlight this potential adverse effect and to discuss the plausible causality of ectopic pregnancy following administration of LNG-only EC.</div>\r\n', '<div>\r', '', 1385454356, 1),
(1015, 1021, 64, 'NATASHA MN ', '', '', '', 1385440149, 0),
(1016, 1022, 64, 'SHENAZ SK', '', '', '', 1385451828, 0),
(1017, 1023, 64, ' AZMA RZ', '', '', '', 1385451829, 0),
(1018, 1024, 64, ' ZARINA AL', '', '', '', 1385451829, 0),
(1019, 1025, 64, ' HAMIDAH A', '', '', '', 1385451829, 0),
(1020, 1026, 64, 'Infantile Acute Megakaryoblastic Leukaemia with T(1:22) in a Non-Down Syndrome Child', '<p>\r\n	Megakaryoblastic leukaemia is the commonest form of leukaemia occuring in Down syndrome infants. However, it&rsquo;s subtype with translocation t(1;22)(p13;q13) is uncommon comprising &lt;1% of all cases and reported to exclusively occur in infant without Down syndrome. It has a female predominance and carries a poor prognosis. We described this rare form of leukaemia in a 9-month-old girl who presented with bruises, massive hepatosplenomegaly and multiple cervical and inguinal lymphadenopathy. The blood film showed severe anaemia with ovalostomatocytosis, thrombocytopenia and mild leucocytosis. The bone marrow aspirate showed numerous blasts showing high nuclear-cytoplasmic ratio and agranular cytoplasm with cytoplasmic blebs. Peroxidase staining was negative. The immunophenotyping of the blasts showed positive expression of CD117, CD13, CD33 and CD61 which confirmed the diagnosis of acute megakaryoblastic leukaemia. Interestingly, the cytogenetic finding of translocation t(1;22) which is most common in acute megaloblastic leukaemia in infants without Down syndrome was found in this case. She received the AML trial 15 ADE protocol chemotherapy regime and developed severe neutropenic sepsis and respiratory distress requiring ventilatory support and granulocyte colony stimulating factor (G-CSF). She recovered well after the first course of chemotherapy and was discharged. Unfortunately, she was not brought in for follow-up chemotherapy and presented a few months later with relapsed AML. She was re-started on ADE protocol and currently is on oral thioguanine for maintenance therapy.</p>\r\n', '<p>\r', '', 1385454595, 1),
(961, 1027, 101, 'Management of Emergency Department Overcrowding (EDOC) in a tertiary teaching hospital', 'Emergency Department Overcrowding (EDOC) has been a longstanding problem. It is defined as a situation where the demand for emergency services exceeds the ability of an Emergency Department (ED) to provide quality care within appropriate time frames.1, 2,  Hospital beds closure or access block to ward admission is one of the most important cause of Emergency s e.g. disaster. A surge response entails even greater responses including implementing Department overcrowding.3  This could be compounded further in events of a patient surge eg affirmative measurement in order to mitigate the issue in tackling the situation. The steps in managing EDOC were: 1.Recognizing EDOC, 2.Initiating action, 3. Maintaining patient flow, 4. Setting clinical goals and 5. Deploying a Surge Team for Advance Triage or Fast Tract', 'Emergency Department Overcrowding (EDOC) has been a longstanding problem. It is defined as a situation where the demand for emergency services exceeds the ability of an Emergency Department (ED) to provide quality care within appropriate time frames.1, 2,  Hospital beds closure or access block to ward admission is one of the most important cause of Emergency s e.g. disaster.', 'changes has been made.\r\nnumber of referrence increased\r\ntitle changed to EDOC - emergency department overcrowding', 1387760728, 1),
(1021, 1028, 108, 'Review of Prospective Study Of Functional Recovery Of Stroke Patients At 3 Months Post Admission: Outcomes And Implications For Post Stroke Care Provision.', '', '', '', 1388443134, 0),
(1022, 1029, 64, 'An Association between Obesity Level and Depression among the Malaysian Working Population', '<div>\r\n	Obesity has become a major health issue in Malaysia. It not only causes a lot of medical problems but also leads to emotional disturbances. Studies have suggested that some degree of depression is prevalent among obese people but the results usually vary. The objective of the present study was to assess the relationship between obesity and depression. A quantitative, cross-sectional study was conducted using a convenient sampling. Respondents who fulfilled the criteria such as the Malaysian working population with a body mass index (BMI) of 30 and above, were randomly selected. A structured questionnaire that consisted of five sections was used to collect data. A total of 60 people participated in the study and the data was analysed using SPSS version 20. The results of the study revealed that the prevalence of depression among the respondents was 23.33%. There was no significant difference in terms of depression between obese male and female (X2= 0.3568, p=0.059). There was association between depression and active measures taken to reduce weight (X2=5.963, p=0.015). The study revealed that not all obese people were depressed but public awareness of the dangers of obesity is important because early detection can prevent unwanted negative outcome among the Malaysian working population.</div>\r\n<div>\r\n	&nbsp;</div>\r\n', '<div>\r', '', 1388715007, 1),
(1023, 1030, 199, 'Tan Chai Eng', '', '', '', 1391913179, 1),
(1024, 1031, 199, 'Noorlaili Mohd Tauhid @ Tohit', '', '', '', 1391913236, 1),
(1025, 1032, 199, 'Lee Chia Ching', '', '', '', 1391913304, 1),
(1026, 1033, 199, 'Mohd Ridzuan Mohd Rani', '', '', '', 1391913347, 1),
(1027, 1034, 199, 'Siti Rahimah Shamshi', '', '', '', 1391913384, 1),
(1028, 1035, 199, 'Ooi Shok Hoon', '', '', '', 1391913436, 1),
(1029, 1036, 199, 'Chronic kidney disease stage 3b among Malaysian diabetics in primary care and its associated factors: a pilot 5-year case control study', 'This study aimed to determine the factors associated with CKD stage 3b among type 2 diabetics attending primary care follow-up, specifically the role of angiotensin blockade dosage.  This was a pilot unmatched case-control study conducted in a teaching primary care centre. Clinical data of 25 cases of diabetic patients with CKD stage 3b (GFR 30-45ml/min/1.73m2) in 2012 were selected for this study, as well as 103 controls who were diabetic patients with GFR more than 45ml/min/1.73m2 in 2012.  Systematic random sampling was employed. Data was obtained from patients’ diabetic records, computerised clinical medical information system and medical case notes. Univariate analysis was done using chi square, t-test, Fisher’s exact test and Mann-Whitney U-test.  Multiple logistic regression was used to determine the associated factors for development of CKD stage 3b.  Cases and controls were different in terms of age, duration of diabetes, use and dosage of angiotensin blockade medications, systolic blood pressure and baseline GFR.   Multiple logistic regression revealed that systolic blood pressure (Adjusted OR= 1.08, 95% CI= 1.02-1.14, p=0.013) and baseline GFR (Adjusted OR= 0.90, 95% CI= 0.85-0.95, p<0.001) was significantly associated with the development of CKD stage 3b among diabetics.  Maximizing the dose of angiotensin blockade had a protective effect (Adjusted OR= 0.14, 95% CI=0.85-0.95, p=0.025)  This study supports the need for good control of systolic blood pressure among diabetic patients to reduce the risk of chronic kidney disease progression.  Dose of angiotensin blockade medications should be optimised in these patients.', 'This study aimed to determine the factors associated with CKD stage 3b among type 2 diabetics attending primary care follow-up, specifically the role of angiotensin blockade dosage.  This was a pilot unmatched case-control study conducted in a teaching primary care centre. Clinical data of 25 cases of diabetic patients with CKD stage 3b (GFR 30-45ml/min/1.73m2) in 2012 were selected for this study, as well as 103 controls who were diabetic patients with GFR more than 45ml/min/1.73m2 in 2012.  Systematic random sampling was employed.', '', 1391913442, 1),
(1030, 1037, 64, 'Vol. 8 No. 1 : 2013', '', '', '', 1392852536, 1),
(1031, 1038, 64, 'DAS S', '', '', '', 1392773331, 0),
(1032, 1039, 64, 'FARIHAH S', '', '', '', 1392773331, 0),
(1033, 1040, 64, 'Palmaris Longus Muscle: Presence or Absence, Does It Matter?', '', '', '', 1392852577, 1),
(1034, 1041, 64, 'SABA ZH', '', '', '', 1392773840, 0),
(1035, 1042, 64, 'Honey: Food or Medicine?', '<p>\r\n	Honey is a natural substance produced by honeybees, Apis mellifera, from the nectar of blossomed flowers or exudates of trees and plants producing nectar honeys or honeydews, respectively. It is a supersaturated solution of sugars, enriched with proteins, minerals, vitamins, organic acids and polyphenols. Honey possesses numerous nutritional, healing and prophylactic properties attributed by the rich components found in honey. Some of the health beneficial properties include wound healing, antimicrobial, antioxidant and anti-inflammatory potential. This review relates the nutritional composition, antioxidant and therapeutical effects of honey with emphasis on Malaysian honeys.</p>\r\n', '<p>\r', '', 1392852599, 1),
(1036, 1043, 64, 'AZMIN S', '', '', '', 1392774670, 0),
(1037, 1044, 64, 'Prospective Study of Functional Recovery of Stroke Patients at Three Months Post Admission: Outcomes and Implications for Post Stroke Care Provision', '<p>\r\n	Functional recovery in the immediate post stroke period predicts the long-term prognosis of post stroke patients. Despite the advancement in stroke rehabilitation in improving the physical function of survivors, there are other factors that may influence functional recovery. We aimed to assess the functional recovery of ischaemic stroke patients attending a tertiary hospital and its associated factors in order to make recommendations for post stroke care after hospital admission. A three months prospective observational study looking at functional recovery using the Modified Barthel&rsquo;s Index (MBI) and depression (Patient Heath Questionnaire-9) score of post stroke patients. There were 46 ischaemic stroke patients who were recruited for the study. At three months, only 37 patients were eligible for analysis. The mean age was 67.2 (SD 11) years with the Malays (50%) making up the majority of the patients, followed by Chinese (41.3%) and Indians (8.7%). There was a total of seven (15.2%) deaths. Hypertension was the most common risk factor (89.1%) followed by dyslipidaemia (65.2%) and diabetes mellitus (63.0%). The mean MBI difference was 45.2 (SD 27.0) with a median MBI of 17.0(IQR 33.0) at baseline compared to 85.0(IQR 42.0) at three months (p &lt; 0.001, CI 35.98,55.45). The prevalence of depression (PHQ-9&ge;10) was 21.6%. Lower functional recovery was found among depressed patients (p &lt;0.026) and among patients with recurrent stroke (p&lt; 0.035). Significant functional recovery can be seen as early as three-months post stroke. Factors such as recurrent stroke and depression may affect functional recovery post stroke; hence should be routinely screened.</p>\r\n', '<p>\r', '', 1392852617, 1),
(1038, 1045, 64, 'RUTH SABRINA S', '', '', '', 1392775022, 0),
(1039, 1046, 64, 'NIK AZLAN NM', '', '', '', 1392775022, 0),
(1040, 1047, 64, 'ADI O', '', '', '', 1392775022, 0),
(1041, 1048, 64, 'Anterior Penetrating Chest Injury with Left Ventricular Thrombus', '<div>\r\n	Urban cities are synonym with a high incidence of penetrating chest injuries either from accidents or interpersonal violence. The outcome of penetrating chest wound can vary from immediate death to a prolonged morbidity. We here report a case of 39-year-gentleman who presented to Emergency Department Hospital Raja Permaisuri Bainun, Ipoh, Perak after being stabbed to the chest. His anterior penetrating chest wound was located at the 5th intercostal space medial to the midclavicular line. The stab wound penetrated the myocardium, causing minimal myocardial rupture. He also suffered from left haemothorax and hemopericardium. The haemothorax was drained with insertion of 32 French chest tube. The patient was admitted under the cardiothoracic team and discharged five days later without surgical intervention. He presented again to the Emergency Department with complains of shortness of breath and pleuritic pain. A left ventricular thrombus was detected via echocardiography. Unfortunately, he took his own discharge. Five days later he came again to Emergency Department with sporadic of loss of vision. The mural thrombus dislodged and embolized to the retinal artery causing amaurosis fugax. The patient was treated with aspirin 150mg and his symptoms subsequently resolved.</div>\r\n', '<div>\r', '', 1392852639, 1),
(1042, 1049, 64, 'SUMATHI U', '', '', '', 1392792356, 0),
(1043, 1050, 64, ' ISMAIL MS', '', '', '', 1392792358, 0),
(1044, 1051, 64, ' AHMAD ZAILANI H', '', '', '', 1392792358, 0),
(1045, 1052, 64, 'LIU CY', '', '', '', 1392792358, 0),
(1046, 1053, 64, 'Case Study on the Effect of Epidural Analgesia on Pain Management During Labour', '<p>\r\n	Child birth is associated with severely painful experience for the parturient, and often exceeds one&rsquo;s expectations. Even though, severe pain is non life-threatening condition in healthy parturient women, it may lead to undesired neuropsychological consequences. When no analgesia was used, postnatal depression may be more common, and this labour pain leads to the development of post-traumatic stress disorder. Epidural analgesia is now considered gold standard for effective pain relief during labour. We here report a case of a 37-year-old G1 P0 patient at term gestation who successfully used epidural analgesia for labour pain management.</p>\r\n', '<p>\r', '', 1392852661, 1),
(1047, 1054, 64, 'MAZLIN MB', '', '', '', 1392792844, 0),
(1048, 1055, 64, 'ADAWIYAH J', '', '', '', 1392792845, 0),
(1049, 1056, 64, 'Homemade Remedies Causing Contact Dermatitis: Case Report on Two Patients with Acne', '<div>\r\n	Natural products are often perceived as safe due to the absence of artificial or chemical materials in its content. However, many are unaware that some of these compounds, albeit natural in existence, can cause harm. Cinnamon and lemon are two commonly used home remedies for acne. Both these naturally existing ingredients are capable of producing dermatitis upon contact with the human skin. The aim of this article is to create awareness among physicians that natural remedies are not free from harm hence, should look out for any possible untoward reactions that these products may cause. Physicians need to explore the possible use of homemade remedies to treat common or minor ailments during history taking as this information may not be given voluntarily. Early identification of the offending agent, adequate management and future avoidance could help prevent further episodes of contact dermatitis and its complications.</div>\r\n', '<div>\r', '', 1392853053, 1),
(1050, 1057, 64, 'AZIZOL M', '', '', '', 1392793166, 0),
(1051, 1058, 64, 'Management of Emergency Department Overcrowding (EDOC) in a Teaching Hospital', '<p>\r\n	Emergency Department Overcrowding (EDOC) has been a longstanding problem. It is defined as a situation where the demand for emergency services exceeds the ability of an Emergency Department (ED) to provide quality care within appropriate time frames. Hospital beds closure or access block to ward admission is one of the most important cause of Emergency s e.g. disaster. A surge response entails even greater responses including implementing Department overcrowding. This could be compounded further in events of a patient surge eg affirmative measurement in order to mitigate the issue in tackling the situation. The steps in managing EDOC were: 1. Recognizing EDOC, 2. Initiating action, 3. Maintaining patient flow, 4. Setting clinical goals and 5. Deploying a Surge Team for Advance Triage or Fast Tract.</p>\r\n', '<p>\r', '', 1392853107, 1),
(1052, 1059, 64, 'WAN HASLINA WAH', '', '', '', 1392793379, 0),
(1053, 1060, 64, 'Masquerade Syndrome: An Ocular Involvement of Lung Cancer', '<p>\r\n	We report a case of a 37-year-old smoker who presented with painless and sudden onset of loss of upper field vision of the right eye, associated with one month history of dry cough which was unresponsive to antibiotic and subsequent anti- tuberculosis therapy. Visual acuity at presentation was counting finger. There was bullous retinal detachment on examination. Radiological imaging revealed multiple lung opacities, involving the brain and the right orbit. Bronchoscopy and biopsy confirmed the diagnosis of adenocarcinoma of the lung. This case illustrates masquerade syndrome of which a lung carcinoma harbours behind a seemingly innocent retinal detachment. It also highlights the importance of entertaining lung carcinoma as a differential diagnosis in suspected tuberculosis among heavy smokers.</p>\r\n', '<p>\r', '', 1392853226, 1),
(1054, 1061, 64, 'Aims and Scope', '<p class="rtejustify">\r\n	<em>Medicine and Health</em> is an open access journal dedicated to publish high quality of scientific research in the field of medicine and allied health sciences. The journal welcomes review articles, original research, case reports and clinical quiz. The journal publishes manuscripts pertaining to any discipline concerned with medicine, dentistry, surgery, nursing, occupational therapy, physiotherapy, biomedical, public health, epidemiological studies and medical education. Authors from any part of the world are welcome to submit their research manuscripts. No publication charges are levied on the submitted manuscript. All articles are published online with free open access.</p>\r\n', '<p class="rtejustify">\r', '', 1433216636, 2),
(1055, 1062, 64, 'Indexation', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	1. Directory of Open Access Journal</p>\r\n<p>\r\n	2. Western Pacific Region Index Medicus</p>\r\n<p>\r\n	3. Chemical Abstracts Service</p>\r\n', '<p>\r\n	&nbsp;</p>\r\n<p>\r\n	1. Directory of Open Access Journal</p>\r\n<p>\r\n	2. Western Pacific Region Index Medicus</p>\r\n<p>\r\n	3. Chemical Abstracts Service</p>\r\n', '', 1433401518, 1),
(1056, 1063, 184, 'Ezalia Esa', '', '', '', 1394006376, 1),
(1057, 1064, 184, 'Elizabeth George', '', '', '', 1394006899, 1),
(1058, 1065, 184, 'Irmi Elfina Ramlan', '', '', '', 1394007516, 1),
(1059, 1066, 184, 'Wan Hayati Mohd Yusoff', '', '', '', 1394008544, 1),
(1060, 1067, 184, 'Chin Yuet Meng', '', '', '', 1394008681, 1),
(1061, 1068, 184, 'Rahimah Ahmad1', '', '', '', 1394008762, 1),
(1062, 1069, 184, 'Nor Asiah Muhamad2', '', '', '', 1394008966, 1),
(1063, 1070, 184, 'Masita Arip', '', '', '', 1394016878, 1),
(1064, 1071, 184, 'Rohaidah Hashim', '', '', '', 1394016961, 1),
(1065, 1072, 184, 'Norhanim Asidin', '', '', '', 1394017792, 1),
(1066, 1073, 184, 'Wahidah Abdullah', '', '', '', 1394159433, 1),
(1067, 1074, 64, 'Thalassaemia Screening Among Healthy Blood Donors In Hospital Tengku Ampuan Rahimah, Klang', '<p>\r\n	ABSTRACT Thalassaemia is a heterogeneous group of genetic disorders with defective synthesis of one or more globin chains. In Malaysia, the most common types are &alpha;-thalassaemia, &beta;-thalassaemia, haemoglobin E and haemoglobin Constant Spring. Detection of these disorders is very important since it may aid in prevention of more serious haemoglobin disorders. We screened 738 healthy blood donors in Hospital Tengku Ampuan Rahimah, Klang from July to September 2010 using cation-exchange high performance liquid chromatography (HPLC). Cases with haemoglobin variants were further analyzed by gel electrophoresis at alkaline pH. Result shows that the blood donors consisted of Malays 370 (56.2%), Indians 139 (21.1%), Chinese 134 (20.4%) and others 13 (2%). There were 16 (2.3%) with haemoglobin E trait, six (0.9%) with co-inheritance of haemoglobin E and &alpha;-thalassaemia and five (0.7%) with &beta;-thalassaemia trait. Haemoglobin Constant Spring and haemoglobin A2 prime were seen in two (0.3%); and Haemoglobin Lepore and alpha chain variant in one (0.2%). &alpha;-thalassaemia and normal haemoglobin A2 &beta;-thalassaemia could not be excluded in 109 cases (16.6%), as they require DNA studies for identification. Thalassaemia screening in blood donors is more feasible and effective. Therefore a wider scale population screening including blood donors could benefit the existing thalassaemia screening programme in Malaysia.</p>\r\n', '<p>\r', '', 1395887733, 1),
(1068, 1075, 101, 'Anuar Deen bin Mohd Kamal', '', '', '', 1394785388, 1),
(1069, 1076, 101, 'Zuraidah Che'' Man', '', '', '', 1394785972, 1),
(1070, 1077, 64, 'STROKE COMMUNITY AWARENESS AMONG PUBLIC: A CROSS SECTIONAL STUDY', '<p>\r\n	This study assesses the public level of knowledge pertaining risk factors, symptoms, treatment and source of information in relation to stroke and transient ischaemic attack. Stroke risk profile is the correlated with the participant&rsquo;s knowledge. Using a validated pre-tested Questionnaire, 112 respondents were recruited during two separate community events. Participants completed the questionnaire and underwent health screening. The data were analysed using SPSS version 20. Results: Overall knowledge was poor. Only 35% of the respondents had satisfactory knowledge of the warning signs (p= 0.94) and 29% had satisfactory knowledge on the risk factors (p=0.46). When divided among risk groups, 26%, 30% and 41% satisfactory awareness were in the low, intermediate and high risk group respectively. None of the respondents knew the availability of treatment of acute stroke in the emergency department or the importance of rehabilitation as part of stroke management. Conclusion: Public awareness on stroke risk factors, symptoms and management is poor.</p>\r\n', '<p>\r', '', 1395885849, 1),
(1071, 1078, 108, 'Review of STROKE COMMUNITY AWARENESS AMONG PUBLIC: A CROSS SECTIONAL STUDY', '', '', '', 1396712890, 0),
(1072, 1079, 228, 'Aqilah-SN SMZ', '', '', '', 1397104487, 1),
(1073, 1080, 228, 'SabarulAM', '', '', '', 1397104577, 1),
(1074, 1081, 228, 'ZulfarinaSM', '', '', '', 1397104665, 1),
(1075, 1082, 228, 'Isa NM', '', '', '', 1397104751, 1),
(1076, 1083, 228, 'NazrunAS', '', '', '', 1397105446, 1),
(1077, 1084, 228, 'Quantitative Ultrasound (QUS) for the assessment of bone health', 'Osteoporosis is known as a silent disease because bone density slowly decreases with advancing age and without symptoms. The incidence of osteoporosis is increasing yearly worldwide. Using conventional bone densitometry to measure bone mineral density (BMD) is standard in the diagnosis of osteoporosis but the cost is high and not practicable in the community. Quantitative ultrasound (QUS) bone measurement is a relatively modern technique for the diagnosis of osteoporosis. It is comparatively easy, reliable, less costly, and a safe method compared to other densitometry techniques. The two QUS parameters currently measured are broadband ultrasound attenuation (BUA) and speed of sound (SOS). QUS also predicts fracture risk independently of BMD. In vitro studies showed that QUS indices were significantly associated with BMD, bone micro architecture and mechanical parameters. In humans, QUS indices were found to be associated with BMD. Hence, QUS is a promising new technique for bone assessment. ', 'Osteoporosis is known as a silent disease because bone density slowly decreases with advancing age and without symptoms. The incidence of osteoporosis is increasing yearly worldwide. Using conventional bone densitometry to measure bone mineral density (BMD) is standard in the diagnosis of osteoporosis but the cost is high and not practicable in the community. Quantitative ultrasound (QUS) bone measurement is a relatively modern technique for the diagnosis of osteoporosis. It is comparatively easy, reliable, less costly, and a safe method compared to other densitometry techniques.', '', 1397105481, 1),
(1078, 1085, 101, 'faizal amri hamzah', '', '', '', 1399277698, 1),
(1079, 1086, 101, 'khairi latiff', '', '', '', 1399277789, 1),
(1080, 1087, 101, 'AUTOMATED EXTERNAL DEFIBRILLATOR (AED) USE AMONG PARAMEDICS IN THE EMERGENCY DEPARTMENT – WHAT ARE THE OBSTACLES IN USING THE AUTOMATED  EXTERNAL DEFIBRILLATOR IN THE PRE-HOSPITAL CARE SETTINGS ?', 'Objective: The aim of this study is to determine and analyse factors that influence paramedics of the Emergency Department of National University Hospital Malaysia in using the AED. This study also assesses their knowledge, confidence levels and identifies areas of deficiencies in AED use.  \r\nDesign: This is a cross sectional prospective study conducted between December 2013 and January 2014. Paramedics from HUKM Emergency Department were enrolled and assessed using the self-filled questionnaire consisting of multiple sections including: knowledge assessment, training and practice.\r\nResult:  53 paramedics participated in this study.  62.3% participants have used AEDs on site. Only 83% participants admit that they would use it if needed. A significant positive correlation is seen between age and work experience with knowledge on AED (p=0.001 and p=0.005 respectively). Government’s institute graduates had better knowledge and higher confidence level than private institutions’ graduates (p<0.001). Statistically significant positive correlations are also seen between working experience and confidence level in deciding to use AED (p=0.006), applying AED (p=0.019) and troubleshooting complications arising from AED use (p=0.002). The main factor reducing confidence level of AED use was lack of training (73.6%) which results in reduced confidence to initiate use (45.3%). 88.6% agreed that training is essential before any AED use. 41.5% felt that AEDs is not ready for Malaysian public.\r\nConclusion: AED usage and knowledge among paramedics are still low. Adequate training is important for paramedics. \r\n', 'Objective: The aim of this study is to determine and analyse factors that influence paramedics of the Emergency Department of National University Hospital Malaysia in using the AED. This study also assesses their knowledge, confidence levels and identifies areas of deficiencies in AED use.  \r\nDesign: This is a cross sectional prospective study conducted between December 2013 and January 2014. Paramedics from HUKM Emergency Department were enrolled and assessed using the self-filled questionnaire consisting of multiple sections including: knowledge assessment, training and practice.\r', '', 1402561934, 1),
(1067, 1088, 184, 'Thalassaemia Screening Among Healthy Blood Donors In Hospital Tengku Ampuan Rahimah, Klang', '         Thalassaemia screening programme has been conducted in Malaysia since 2004. The aim of the programme is to reduce the burden of the disease by identifying thalassaemia carriers. However, the response towards the screening activities was unsatisfactory as there was a lack of public awareness against the importance of thalassaemia screening. An alternative approach is to screen blood donors. The purpose of this study was to find out the prevalence of thalassaemia carriers among healthy blood donors.\r\n	Seven hundred and thirty eight healthy blood donors were screened in Hospital Tengku Ampuan Rahimah, Klang from July to September 2010 using cation-exchange high performance liquid chromatography (HPLC). Cases with haemoglobin variants were further analyzed by gel electrophoresis at alkaline pH. \r\n        Result shows that the blood donors consisted of Malays 413 (56%), Indians 162 (22%), Chinese 148 (20%) and others 15 (2%). There were  19 (2.6%) with haemoglobin E trait, six (0.8%) with co-inheritance of haemoglobin E and α-thalassaemia and five (0.7%) with β-thalassaemia trait. Haemoglobin Constant Spring and haemoglobin A2 prime were seen in two (0.3%); and Haemoglobin Lepore and alpha chain variant in one (0.2%). α-thalassaemia and normal haemoglobin A2 β-thalassaemia could not be excluded in 190 cases (26%), as they require DNA studies for identification. \r\n        Thalassaemia screening in blood donors is more feasible and effective. Therefore a wider scale population screening including blood donors could benefit the existing thalassaemia screening programme in Malaysia.\r\n\r\n\r\n\r\n\r\n\r\n\r\n', '         Thalassaemia screening programme has been conducted in Malaysia since 2004. The aim of the programme is to reduce the burden of the disease by identifying thalassaemia carriers. However, the response towards the screening activities was unsatisfactory as there was a lack of public awareness against the importance of thalassaemia screening. An alternative approach is to screen blood donors. The purpose of this study was to find out the prevalence of thalassaemia carriers among healthy blood donors.\r', 'Revised manuscript', 1401469815, 1),
(1081, 1089, 236, 'Sundaresan A N', '', '', '', 1402646031, 1),
(1082, 1090, 236, 'Vikram Mohan', '', '', '', 1402646148, 1),
(1083, 1091, 236, 'Nur Farhana Mohd Yunus', '', '', '', 1402646203, 1),
(1084, 1092, 236, 'Mohandas kurup', '', '', '', 1402646273, 1),
(1085, 1093, 236, 'Potturi Gowri Shankar', '', '', '', 1402648284, 1),
(1086, 1094, 236, 'Mahendran J', '', '', '', 1402648336, 1),
(1087, 1095, 236, 'Impact of Total Contact Plaster Boot via off loading practice In Trophic Ulcers of the Feet: Case series.', 'Trophic ulcers have emerged as one of the major complications following Diabetes Mellitus and Hansen’s diseases. In this case series, the study attempted total contact plaster boot using a readily available plaster of Paris to treat trophic ulcer for 10 subjects. A total of 5 subjects with Diabetes Mellitus and 5 subjects with Hansen’s diseases were included based on the study criteria. Pre and post test measure of wound measurement size following total contact plaster boot were taken as an outcome measure. All ten subjects showed decrease in size of wound following fifteen days of treatment. No adverse effects associated with this type of treatment. Subjects with trophic ulcer may benefit from the application of total contact plaster boot.  ', 'Trophic ulcers have emerged as one of the major complications following Diabetes Mellitus and Hansen’s diseases. In this case series, the study attempted total contact plaster boot using a readily available plaster of Paris to treat trophic ulcer for 10 subjects. A total of 5 subjects with Diabetes Mellitus and 5 subjects with Hansen’s diseases were included based on the study criteria. Pre and post test measure of wound measurement size following total contact plaster boot were taken as an outcome measure. All ten subjects showed decrease in size of wound following fifteen days of treatment.', '', 1402648465, 1),
(1088, 1096, 64, 'Vol. 8 No. 2 : 2013', '', '', '', 1405043193, 1),
(1089, 1097, 64, 'Kosai NR', '', '', '', 1405038290, 0),
(1090, 1098, 64, 'Is Bariatic Surgery The Cure for Diabetes Mellitus?', '', '', '', 1405043005, 1),
(1091, 1099, 64, 'TAN CE', '', '', '', 1405038721, 0),
(1092, 1100, 64, 'TOHIT N', '', '', '', 1405038721, 0),
(1093, 1101, 64, 'LEE CC', '', '', '', 1405039827, 0),
(1094, 1102, 64, 'MOHD RIDZUAN AR', '', '', '', 1405039827, 0),
(1095, 1103, 64, 'SITI RAHIMAH S', '', '', '', 1405039827, 0),
(1096, 1104, 64, 'OOI SH', '', '', '', 1405039828, 0),
(1097, 1105, 64, 'Chronic Kidney Disease Stage 3B among Malaysian Diabetics in Primary Care and its Associated Factors: A Pilot 5-Year Case Control Study', '<p>\r\n	The present study aimed to determine the factors associated with CKD stage 3b among type 2 diabetics attending primary care follow-up, specifically the role of angiotensin blockade dosage. This was a pilot unmatched case-control study conducted in a teaching primary care centre. Clinical data of 25 cases of diabetic patients with CKD stage 3b (GFR 30-45ml/min/1.73m2) in 2012 were selected for this study, as well as 103 controls who were diabetic patients with GFR more than 45ml/min/1.73m2 in 2012. Systematic random sampling was employed. Data was obtained from patients&rsquo; diabetic records, computerised clinical medical information system and medical case notes. Univariate analysis was done using Chi-square, t-test, Fisher&rsquo;s exact test and Mann-Whitney U-test. Multiple logistic regression was used to determine the associated factors for development of CKD stage 3b. Cases and controls were different in terms of age, duration of diabetes, use and dosage of angiotensin blockade medications, systolic blood pressure and baseline GFR. Multiple logistic regression revealed that systolic blood pressure (Adjusted OR= 1.08, 95% CI= 1.02-1.14, p=0.013) and baseline GFR (Adjusted OR= 0.90, 95% CI= 0.85-0.95, p&lt;0.001) was significantly associated with the development of CKD stage 3b among diabetics. Maximizing the dose of angiotensin blockade had a protective effect (Adjusted OR= 0.14, 95% CI=0.85-0.95, p=0.025). The results of the present study supports the need for good control of systolic blood pressure among diabetic patients to reduce the risk of chronic kidney disease progression. Dose of angiotensin blockade medications should be optimised in these patients.</p>\r\n', '<p>\r', '', 1405043022, 1),
(1098, 1106, 64, 'SYUHADA N', '', '', '', 1405040452, 0),
(1099, 1107, 64, 'TZAR MN', '', '', '', 1405040452, 0),
(1100, 1108, 64, 'RAMLIZA R', '', '', '', 1405040453, 0),
(1101, 1109, 64, 'Clostridium difficile Infection: Clinico-Epidemiological Perspective', '<p>\r\n	Clostridium difficile infection (CDI) causes mild to severe diarrhoea and pseudomembranous colitis in patients who had prior antibiotic exposure. Despite CDI being prevalent worldwide, its epidemiological data is scanty in Malaysia. This study aimed to determine the prevalence and incidence of CDI at Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Stool specimens from 147-suspected CDI patients were obtained from 1 November 2011 until 31 October 2012. The presence of C. difficile toxin A and/or B were detected using a commercial immunochromatographic kit (Wampole&trade; Tox A/B Quik Chek). Surveillance data was collected from patients&rsquo; medical records to establish the demographic and clinical characteristics. The overall prevalence and incidence of CDI in UKMMC was 6.1% and 5.2 cases per 10 000 patient-days, respectively. Among nine CDI patients, 77.8% were males and 55.6% were Chinese. CDI was most common in medical wards (88.9%). The median age was 60 years and the median length of hospital stay was 13 days. Majority (88.9%) of CDI patients received antibiotics eight weeks prior to CDI. Penicillin-beta-lactamase inhibitors were the most common antecedent antibiotics. Five (55.6%) CDI patients received acid suppressant medications. The in-hospital mortality rate was 22.2%. In conclusion, the prevalence and incidence of CDI at UKMMC is relatively low and occurs sporadically.</p>\r\n', '<p>\r', '', 1405043038, 1),
(1102, 1110, 64, 'KOO YL', '', '', '', 1405040890, 0),
(1103, 1111, 64, 'HING HL', '', '', '', 1405040890, 0),
(1104, 1112, 64, 'WIDAD O', '', '', '', 1405040891, 0),
(1105, 1113, 64, 'CHUNG HT', '', '', '', 1405040891, 0),
(1106, 1114, 64, 'NABISHAH M', '', '', '', 1405040891, 0),
(1107, 1115, 64, 'HO CCK', '', '', '', 1405040891, 0),
(1108, 1116, 64, 'Clinical Decision Making Ability of Nursing Students in a Tertiary Hospital', '<p>\r\n	Decision making in nursing is one of the most important skills nurses must apply and utilize in their nursing practice. The aim of this study was to determine the perception of clinical decision making ability among nursing students. A descriptive cross-sectional study was conducted in a tertiary hospital. A total of 54 nursing students were recruited using a modified version of Clinical Decision Making in Nursing Scale (CDMNS) adapted from Jenkins (1985). The findings showed good CDMNS score with mean and standard deviation of (124.24&plusmn;12.713). The four sub-scales of CDMNS were: searching for alternative (33.24&plusmn;4.821), canvassing (28.74&plusmn;3.514), evaluation and re-evaluation (31.43&plusmn;3.922), searching for information (30.83&plusmn;4.765). Nineteen (35%) of the participants chose nursing as their first choice, whereas 35 participants (65%) did not. Thirthy seven (69%) participants were satisfied with their nursing competency, 17 (31%) were unsatisfied. There were significant differences between searching for alternatives, evaluation and re-evaluation, and nursing as their first choice (p=&lt;0.05). There were also significant differences between searching for alternatives and satisfaction with nursing competency (p=&lt;0.05). There was significant difference between education level and searching for alternatives and information (p=&lt;0.05). The nursing students possessed adequate clinical decision making ability. Although most of the nursing students did not choose nursing as their first choice, they sought for alternatives and evaluated-reevaluated during their decision making process. Nursing students&rsquo; satisfaction also contributed to appropriate clinical decision making in the critical care setting.</p>\r\n', '<p>\r', '', 1405470923, 1),
(1109, 1117, 64, 'BALAKRISHNIAN M', '', '', '', 1405041179, 0),
(1110, 1118, 64, 'AHMAD KHALDUN I', '', '', '', 1405041179, 0),
(1111, 1119, 64, 'HAMIDAH Y', '', '', '', 1405041179, 0),
(1112, 1120, 64, 'Paramedics’ Knowledge on Intraosseous Cannulation', '<p>\r\n	Access to an intravenous (IV) route is very crucial in emergency patients under resuscitation. The difficulty to access and administer fluid and drugs through intravenous will influence the outcome of patient. In case of unavailable of intravenous route, the alternative is intraosseous access. To date, there is no data available on the prevalence and pattern of intraosseous cannulation knowledge among emergency paramedics in the published literature from our country, even though the use of intraosseous cannulation for emergency patients is being practices. Therefore, the purpose of the present study was to determine the level of knowledge regarding intraosseous cannulation among emergency paramedics. The knowledge related to intraosseous cannulation among emergency paramedics was assessed through structured validated test questions. Fifteen paramedics participated in this study. Majorities of participants were male (86.6%). The age group ranged from 22 &ndash; 45 old years. The working experiences were from two to thirteen years. There was only 1 out of 15 participants who had scored 75%. The majority (10) scored 40% to 50%. This suggests that necessity in teaching of intraosseous cannulation among emergency paramedics needs to be emphasised.</p>\r\n', '<p>\r', '', 1405043073, 1),
(1113, 1121, 64, 'FAZARINA M', '', '', '', 1405041462, 0),
(1114, 1122, 64, 'Tuberculosis of the Prostate Presenting as Benign Prostatic Hyperplasia', '<p>\r\n	Genitourinary system is the second most common site of tuberculosis (TB) after pulmonary system. Tuberculosis of the prostate is a rare manifestation of genitourinary tuberculosis. It is even more uncommon if occurring in an immune-competent individual. Prostate TB is usually an incidental finding in transurethral resection of prostate (TURP). Here, we report a case of TB of the prostate in a 70-year-old man who had benign prostatic hyperplasia for two years. TB was diagnosed incidentally post-TURP. In this report, we also discuss additional tests which may help in establishing the diagnosis of genitourinary tuberculosis.</p>\r\n', '<p>\r', '', 1405043082, 1),
(1115, 1123, 64, 'REZA MQ', '', '', '', 1405041738, 0),
(1116, 1124, 64, 'What Eyes Behold if Missed can be a Life Taking Event: A Case of Orbital Cellulitis', '<p>\r\n	Orbital cellulits is one of the life threatening event that should not be missed out and must be distinguished from preseptal cellulitis. It is an infective process involving ocular adnexal structures posterior to the orbital septum. High index of suspicion is the key to its diagnosis as even experienced physician can miss. Here, we present a case of a 15-year-old female who presented with progressive swelling over right forehead associated with high grade fever, headache and purulent discharge of the swelling. She was diagnosed with sepsis due to right forehead abscess and was treated with intravenous antibiotics followed with admission. However, she suddenly deteriorated in the ward which required intubation and thereby Intensive Care Unit (ICU) admission. Investigations revealed fluid collection at right retrobulbar space suggestive of an abscess where emergency drainage was carried out. Unfortunately, her condition worsened and patient succumbed at day-10 of admission, despite all efforts.</p>\r\n', '<p>\r', '', 1405043097, 1),
(1117, 1125, 64, 'NARISA SS', '', '', '', 1405042103, 0),
(1118, 1126, 64, 'SHANTI P', '', '', '', 1405042103, 0),
(1119, 1127, 64, 'JEEVINESH NA', '', '', '', 1405042103, 0),
(1120, 1128, 64, 'SAKTHISWARY R', '', '', '', 1405042104, 0),
(1121, 1129, 64, 'Eosinophilic Gastroenteritis as the Initial Manifestation of Hypereosinophilic Syndrome', '<p>\r\n	Eosinophilic gastroenteritis, an inflammatory disease of unknown etiology, commonly involves the stomach and small intestine with eosinophilic infiltration. Here, we report an unusual case of eosinophilic gastroenteritis involving the entire digestive tract as a manifestation of hypereosinophilic syndrome (HES). A 22-year-old woman presented to us with diarrhoea, pleural effusion, ascites and marked peripheral oeosinophilia. Stool specimens were negative for parasites, ova, bacteria, and fungi. Endoscopic studies showed pangastritis and duodenitis. Biopsy specimens of the oesophagus, stomach, duodenum, ileum, and colon demonstrated oeosinophilic infiltration. A diagnosis of hypereosinophilic syndrome with eosinophilic gastroenteritis involving the entire digestive tract was made. Hence, she was treated with prednisolone. Symptoms and peripheral oeosinophilia rapidly resolved with treatment, and radiological investigations revealed resolution of effusion. This case illustrates the wide spectrum of clinical manifestation of the disease, whereby it involves the entire digestive tract and it also emphasizes the diagnostic yields of endoscopic biopsies.</p>\r\n', '<p>\r', '', 1405043115, 1),
(1122, 1130, 64, 'SAMANTA PP', '', '', '', 1405042395, 0),
(1123, 1131, 64, 'KHARB P', '', '', '', 1405042395, 0),
(1124, 1132, 64, 'ATULYA', '', '', '', 1405042396, 0),
(1125, 1133, 64, 'Variant Origin of Common Hepatic and Middle Colic Artery-A Case Report', '<p>\r\n	Variation of common hepatic or middle colic artery may account for the complications which arise during liver transplantation and surgeries of upper abdomen. We here report a case of an anomalous branching pattern of ventral branches of abdominal aorta in a 50-year-old male cadaver. The coeliac trunk originated from the ventral aspect of abdominal aorta and bifurcated into left gastric and splenic arteries. The common hepatic artery originated directly from the abdominal aorta, 0.2cm inferior to the origin of coeliac trunk. The middle colic artery originated from the proximal segment of splenic artery instead of its usual origin, the superior mesenteric artery. A separate middle colic branch of superior mesenteric artery was not observed. Anatomical variations in the branching patterns of coeliac trunk and superior mesenteric arteries are not infrequent. However, the concomitant variant origins of branches of coeliac trunk and middle colic artery is a rare anomaly and its knowledge would therefore be helpful in operative procedures of abdomen and image guided interventions.</p>\r\n', '<p>\r', '', 1405043123, 1),
(1126, 1134, 97, 'Christopher Ho Chee Kong', '', '', '', 1405234083, 1),
(1127, 1135, 97, 'Dariah Mohd Yusoff', '', '', '', 1405234143, 1),
(1128, 1136, 97, ' Lily Xiao', '', '', '', 1405234184, 1),
(1129, 1137, 97, ' Ingrid Belan', '', '', '', 1405234209, 1),
(1130, 1138, 97, ' Jan Paterson', '', '', '', 1405234237, 1),
(1131, 1139, 97, 'Postnatal Urinary Incontinence: Prevalence and Factors Associated with it in a Malaysian Population', 'The aim of this study was to investigate the prevalence of postnatal urinary incontinence (PNUI), the risk factors associated with it and postnatal women’s (PN) level of engagement in practicing traditional Malay confinement care (TMCC) in Kelantan, Malaysia. A cross-sectional survey was conducted among 362 postnatal women aged between 18 – 45 years and who were between three to five months post-delivery. Participants were selected through convenience sampling at six Maternal and Child Health Clinics located in comparatively similar districts. Descriptive and inferential analyses including, factor analysis, Pearson chi-square, Mann-Whitney U test and logistic regressions analysis were used for data analysis.  Overall, PNUI was reported by 80 women (22.1%). Vaginal delivery was associated with PNUI. All PN women within the study context practiced TMCC. The findings revealed that women who had PNUI were more likely to engage in a higher level of the ‘Body care’ component of TMCC. UI is prevalent among PN women in Kelantan, Malaysia. Further research into the impact of the ‘body care’ component of TMCC on PNUI is required. ', 'The aim of this study was to investigate the prevalence of postnatal urinary incontinence (PNUI), the risk factors associated with it and postnatal women’s (PN) level of engagement in practicing traditional Malay confinement care (TMCC) in Kelantan, Malaysia. A cross-sectional survey was conducted among 362 postnatal women aged between 18 – 45 years and who were between three to five months post-delivery. Participants were selected through convenience sampling at six Maternal and Child Health Clinics located in comparatively similar districts.', '', 1405234378, 1),
(1132, 1140, 101, 'hilwani kaharuddin', '', '', '', 1407223671, 1),
(1133, 1141, 101, 'heng miao ching', '', '', '', 1407223866, 1),
(1134, 1142, 101, 'Syazwani Azwa', '', '', '', 1407223959, 1),
(1135, 1143, 101, 'Qamarul Ariffin Samin', '', '', '', 1407224043, 1),
(1136, 1144, 101, 'nazurah bt md shah', '', '', '', 1407224681, 1),
(1137, 1145, 101, 'ADVERSE DRUG REACTION WITH MIDAZOLAM USE IN EMERGENCY DEPARTMENT', '<p>\r\n	Midazolam is one of the most commonly used drugs for sedation in Emergency Department.This is a retrospective study conducted on 380 patients from December 2012 to May 2014 in Emergency Department (ED) UKMMC. The objective is to elicit the frequency of side effects and correlation to various factors i.e socio-demography, co-morbidities, age groups and underlying illnesses. RESULTS: Out of 380 patients, 35 patients had side effects (20 patients with midazolam alone, 15 patients with combination of drugs) The average age was 42 years and the average dose of midazolam was 3.5mg. The most common other drug combined was Fentanyl. The overall complication rate for midazolam was 5.3%. The most common side effect recorded was excessive somnolence (1.6%). Other side effects were local skin reaction (1.1%), vomiting (0.8%), headache (0.8%) and hypotension (0.5%). There was no significant association between the socio-demographic factors and drugs combination with the side effects of midazolam on patients. It was concluded from this study that midazolam is a very safe drug due to no life threatening side effects found. There are possibilities that most side effects recorded could be caused by other compound factors for example underlying injuries or disease and combination with other drugs.</p>\r\n', '<p>\r', '', 1432890836, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(1138, 1146, 266, 'Tan Toh Leong', '', '', '', 1408351187, 1),
(1139, 1147, 266, 'Noor Akmal Shareela Ismail ', '', '', '', 1408351312, 1),
(1140, 1148, 266, 'Noor Asmidar binti Anuar ', '', '', '', 1408352395, 1),
(1141, 1149, 266, 'Ahmad Fuad Fahmi bin Mohd Noor ', '', '', '', 1408352457, 1),
(1142, 1150, 266, 'Ong Wei Jie ', '', '', '', 1408352576, 1),
(1143, 1151, 266, 'Chieng Zhin Liang', '', '', '', 1408352757, 1),
(1144, 1152, 266, ' 28-day mortality prediction in sepsis patients using static lactate concentration and early lactate clearance: An observational study', 'Sepsis causes high mortality and morbidity. Static lactate concentration and early lactate clearance are cited to be a predictor for sepsis survival. This study examines the clinical utility of static lactate concentration and early lactate clearance within the first 6 hours of admission in Emergency Department (ED) to predict 28-day mortality rate in sepsis patients. Patients who presented with sepsis, severe sepsis or septic shock and admitted to ED of Universiti Kebangsaan Malaysia Medical Centre were recruited. Blood lactate concentrations were measured upon admission (H0), at 1st hour (H1) and 6th hour (H6) respectively. Either standard treatment of sepsis or early goal directed therapy was initiated according to sepsis severity. A follow-up report was conducted at 28 days via telephone call, e-mail or case notes. Patients were later classified into survivor and non-survivor as final outcome. Static lactate concentration appeared to be significantly higher for non-survivor as compared to the survival group at H0, H1 and H6 (p<0.05). The lactate clearance trend reflects no relationship between early lactate clearance and 28-day mortality. Static lactate concentration showed a superior predictor for sepsis over early lactate clearance. Although early lactate clearance was unable to prove its ability to predict 28-day mortality, our findings suggest it can be a useful tool to gauge the resuscitation outcome.', 'Sepsis causes high mortality and morbidity. Static lactate concentration and early lactate clearance are cited to be a predictor for sepsis survival. This study examines the clinical utility of static lactate concentration and early lactate clearance within the first 6 hours of admission in Emergency Department (ED) to predict 28-day mortality rate in sepsis patients. Patients who presented with sepsis, severe sepsis or septic shock and admitted to ED of Universiti Kebangsaan Malaysia Medical Centre were recruited.', '', 1418109640, 1),
(1145, 1153, 236, 'Vengatasubramani Manoharan', '', '', '', 1410844001, 1),
(1146, 1154, 236, 'Physical Training Improved Cardiovascular fitness level among Chronic Obstructive Pulmonary Disease Patients', 'Chronic obstructive pulmonary disease (COPD) patients reported to have cardiovascular instability which leads to greater limitation for activities there by leads to poor quality of life. Physical training proved to be one of the moderators of these limitations. However, uncertainty prevails among the protocol and duration.  The present study investigated the effect of physical training on blood pressure, heart rate and Rate pressure Product (RPP) among COPD patients. A total of thirty COPD patients who are aged between 40 to 55 years old were recruited for the study based on the inclusion and exclusion criteria and were assigned in to experimental group (15 patients) and control group (15 patients). The mean difference of blood pressure, heart rate and RPP were analyzed using paired t-test. There was significant different between the pre and post test values of all parameters between experimental and control group with p<0.05. Diastolic blood pressure showed to be less significant as compared to systolic blood pressure. This showed that specifically designed physical improved the cardiovascular fitness among COPD patients. ', 'Chronic obstructive pulmonary disease (COPD) patients reported to have cardiovascular instability which leads to greater limitation for activities there by leads to poor quality of life. Physical training proved to be one of the moderators of these limitations. However, uncertainty prevails among the protocol and duration.  The present study investigated the effect of physical training on blood pressure, heart rate and Rate pressure Product (RPP) among COPD patients.', '', 1410845843, 1),
(1147, 1155, 275, 'Ahmad Nabil Md Rosli', '', '', '', 1411722681, 1),
(1148, 1156, 275, 'Azlin Baharudin', '', '', '', 1411722727, 1),
(1149, 1157, 275, 'Managing ‘Schizophrenia In the Midst of Schizophrenia’: The Role of Assertive Community Treatment ', 'This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with 2 other family members with the same illness and their carer who developed major depression from shouldering the burdens of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.  ', 'This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with 2 other family members with the same illness and their carer who developed major depression from shouldering the burdens of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.  ', '', 1417592821, 1),
(1150, 1158, 101, 'Mohd Fairuz bin Ali', '', '', '', 1413451436, 1),
(1151, 1159, 101, 'Premalatha Subramaniam', '', '', '', 1413453061, 1),
(1152, 1160, 101, 'Orogastric lavage for fatal poisoning: A lifesaving procedure now forgotten', 'Orogastric lavage has been performed since 200 years ago for intoxicated patients. Due to the risk that outweighs benefits it has fallen out of favour for the last decade. Presented to out emergency department doorstep was a teenage girl who ingested a bottle of pesticide within the time frame before gastric emptying. The girl was resuscitated, intubated and orogastric lavage was performed. 50 cc of the toxic substance was siphoned and antidote of the toxin was administered. She was admitted to the intensive care unit and subsequently recovered to discharge 5 days later. Definitive airway management, proper technique, correct selection of patients and adequate monitoring are paramount to the success of orogastric lavage. ', 'Orogastric lavage has been performed since 200 years ago for intoxicated patients. Due to the risk that outweighs benefits it has fallen out of favour for the last decade. Presented to out emergency department doorstep was a teenage girl who ingested a bottle of pesticide within the time frame before gastric emptying. The girl was resuscitated, intubated and orogastric lavage was performed. 50 cc of the toxic substance was siphoned and antidote of the toxin was administered. She was admitted to the intensive care unit and subsequently recovered to discharge 5 days later.', '', 1413453354, 1),
(1153, 1161, 387, 'Noor Dina Hashim', 'Date of birth : 22nd April 1981\r\nUndergraduate : 2005 UKM\r\nKKM Service : 2005-2008 GH Malacca\r\nTrainee Lecturer (SLAB) program UKM : 2008\r\nPostgraduate ORL-HNS UKM : 2009-2013\r\nCurrently lecturer & ORL-HNS specialist in UKMMC', 'Date of birth : 22nd April 1981\r\nUndergraduate : 2005 UKM\r\nKKM Service : 2005-2008 GH Malacca\r\nTrainee Lecturer (SLAB) program UKM : 2008\r\nPostgraduate ORL-HNS UKM : 2009-2013\r\nCurrently lecturer & ORL-HNS specialist in UKMMC', '', 1416443444, 1),
(1154, 1162, 64, 'THE RARE PRIMARY LOW GRADE PAPILLARY ADENOCARCINOMA OF NASOPHARYNX ; A DIAGNOSTIC CHALLENGE AND MANAGEMENT', '<p>\r\n	Low Grade Papillary Nasopharyngeal Adenocarcinoma (LGPAC) is a very rare tumour of mucosal origin as compared to a higher incident of well differentiated keratinized/non keratinized nasopharyngeal carcinoma. It is an epithelial tumour with glandular differentiation. Its papillary figure seen histologically, is also seen in metastatic papillary thyroid carcinoma. This has caused a significant challenge to the pathologist to differentiate primary papillary nasopharyngeal adenocarcinoma and metastatic tumour. Thyroid Transcription Factor-1 (TTF-1) is also expressed in subsets of papillary nasopharyngeal adenocarcinoma, which is valuable as a diagnostic tool. LGPAC is a benign-like malignant neoplasm. An early diagnosis with a complete tumour removal via conventional excision or endoscopic approach has offered a good prognosis with low risk of recurrence.</p>\r\n', '<p>\r', '', 1421120710, 1),
(1155, 1163, 64, 'Vol. 9 No. 1 : 2014', '', '', '', 1417664140, 1),
(1156, 1164, 64, 'ZULFARINA SM', '', '', '', 1417596420, 0),
(1157, 1165, 64, 'SABARUL AM', '', '', '', 1417596421, 0),
(1158, 1166, 64, 'Quantitative Ultrasound (QUS) for the Assessment of Bone Health', '<div>\r\n	Osteoporosis is known as a silent disease because bone density slowly decreases with advancing age and without symptoms. The incidence of osteoporosis is increasing yearly worldwide. Measuring bone mineral density (BMD) using conventional bone densitometry (DXA) is practical in diagnosis of osteoporosis but the cost is high and cannot be implemented in community. However, quantitative ultrasound (QUS) is a modern technique to measure the bone density and also for the diagnosis of osteoporosis. It is comparatively easy, reliable, less costly, and a safe method compared to other techniques. QUS consists of two main parameters which are known as broadband ultrasound attenuation (BUA) and speed of sound (SOS). QUS can also predict fracture risk of BMD. QUS showed significantly associated with BMD, bone micro architecture and mechanical parameters for In vitro studies and in human studies, QUS were found to be associated with BMD. Hence, QUS is capable to be new technique for bone assessment.</div>\r\n', '<div>\r', '', 1417596523, 1),
(1159, 1167, 64, 'SITI ZUBAIDAH M', '', '', '', 1417597029, 0),
(1160, 1168, 64, 'NOR HIDAYATI S', '', '', '', 1417597029, 0),
(1161, 1169, 64, 'NOOR FARISAH AR', '', '', '', 1417597029, 0),
(1162, 1170, 64, 'NOOR HAMIDAH H', '', '', '', 1417597030, 0),
(1163, 1171, 64, 'AINOON O', '', '', '', 1417597030, 0),
(1164, 1172, 64, 'Detection of Partial G6PD Deficiency using OSMMR2000-D Kit with Hb Normalization', '<p>\r\n	Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzyme deficiency worldwide including Malaysia. Screening of cord blood for partial G6PD deficiency is important as they are also prone to develop acute haemolysis. In this study, we determined the prevalence of partial G6PD deficient in paediatric population aged 1 month-12 years and normal term female neonates using OSMMR-D kit with haemoglobin (Hb) normalization and compare it with florescence spot test (FST). A total of 236 children, aged between between 1 month-12 years and 614 normal term female neonates were recruited for this study. Determination of normal means for G6PD activity and; cut-off points for partial and severe deficiency were determined according to WHO Working Group (1989). Determination of prevalence for partial deficiency for both groups (female patient) was done using this enzyme assay kit and findings were compared with FST. In this study, 15.7% (18/115) female children were classified as partial G6PD deficient by quantitative enzyme method (G6PD activity: 4.23-5.26U/gHb). However, FST only detected 0.9% (1/115) with minimal G6PD activity. The prevalence of partial G6PD deficiency in female neonate group was 3.42% (21/614) by enzyme assay versus 0.49% (3/614) by FST. This study concluded that our routine screening method using FST was unable to diagnose female heterozygotes. We recommend using this quantitative enzyme assay method by OSMMR-D kit since it was more sensitive in detecting G6PD deficiency in female neonates compared to FST.</p>\r\n', '<p>\r', '', 1417663511, 1),
(1165, 1173, 64, 'DARIAH MY1, 2, I2, 2, 3,  4', '', '', '', 1417597584, 0),
(1166, 1174, 64, 'LILY X', '', '', '', 1417597585, 0),
(1167, 1175, 64, 'PATERSON J', '', '', '', 1417597585, 0),
(1168, 1176, 64, 'CHRISTOPHER CK HO', '', '', '', 1417597585, 0),
(1169, 1177, 64, 'Postnatal Urinary Incontinence: Prevalence and Factors Associated with It in a Malaysian Population', '<p>\r\n	The aim of this study was to investigate the prevalence of postnatal urinary incontinence (PNUI), the risk factors associated with it and postnatal women&rsquo;s (PN) level of engagement in practicing traditional Malay confinement care (TMCC) in Kelantan, Malaysia. A cross-sectional survey was conducted among 362 postnatal women aged between 18 &ndash; 45 years and who were between three to five months post-delivery. Participants were selected through convenience sampling at six Maternal and Child Health Clinics located in comparatively similar districts. Descriptive and inferential analyses including, factor analysis, Pearson chi-square, Mann-Whitney U test and logistic regressions analysis were used for data analysis. Overall, PNUI was reported by 80 women (22.1%). Vaginal delivery was associated with PNUI. All PN women within the study context practiced TMCC. The findings revealed that women who had PNUI were more likely to engage in a higher level of the &lsquo;Body care&rsquo; component of TMCC. UI is prevalent among PN women in Kelantan, Malaysia. Further research into the impact of the &lsquo;body care&rsquo; component of TMCC on PNUI is required.</p>\r\n', '<p>\r', '', 1417663837, 1),
(1170, 1178, 64, 'DARIAH MY', '', '', '', 1417597768, 0),
(1171, 1179, 64, 'ANUAR DEEN MK', '', '', '', 1417598552, 0),
(1172, 1180, 64, 'MOHD FAIRUZ A', '', '', '', 1417598552, 0),
(1173, 1181, 64, 'ZURAIDAH CM', '', '', '', 1417598552, 0),
(1174, 1182, 64, 'Stroke Community Awareness among Public: A Cross Sectional Study', '<div>\r\n	This study assessed the level of public awareness pertaining to risk factors, symptoms, treatment and source of information in relation to stroke and transient ischaemic attack. Stroke risk profile of the respondents was correlated with their level of knowledge. Using a validated pre-tested questionnaire, 112 respondents were recruited during two separate community events. Respondents completed the questionnaire and underwent health screening. The data were analysed using SPSS version 20. Overall knowledge was poor. Only 35% of the respondents had satisfactory knowledge of the warning signs (p= 0.94) and 29% had satisfactory knowledge on the risk factors (p=0.46). When analysed according to risk groups, 26%, 30% and 41% of respondents had satisfactory awareness in the low, intermediate and high risk group, respectively. None of the respondents knew about the availability of treatment of acute stroke in the emergency department or the importance of rehabilitation as part of stroke management. Public awareness on stroke risk factors, symptoms and management is poor.</div>\r\n', '<div>\r', '', 1417663867, 1),
(1175, 1183, 64, 'EZALIA E', '', '', '', 1417652446, 0),
(1176, 1184, 64, 'IRMI ELFINA R', '', '', '', 1417652448, 0),
(1177, 1185, 64, ' ELIZABETH G', '', '', '', 1417652449, 0),
(1178, 1186, 64, 'WAN HAYATI MY', '', '', '', 1417652449, 0),
(1179, 1187, 64, 'NORHANIM A', '', '', '', 1417652449, 0),
(1180, 1188, 64, 'WAHIDAH A', '', '', '', 1417652449, 0),
(1181, 1189, 64, 'CHIN YM', '', '', '', 1417652449, 0),
(1182, 1190, 64, 'RAHIMAH A', '', '', '', 1417652450, 0),
(1183, 1191, 64, 'NOR ASIAH M', '', '', '', 1417652450, 0),
(1184, 1192, 64, 'MASITA A', '', '', '', 1417652450, 0),
(1185, 1193, 64, 'ROHAIDAH H', '', '', '', 1417652450, 0),
(1186, 1194, 64, 'Thalassaemia Screening among Healthy Blood Donors in Hospital Tengku Ampuan Rahimah, Klang', '<p>\r\n	Thalassaemia screening programme has been conducted in Malaysia since 2004. The aim of the programme was to reduce the burden of the disease by identifying thalassaemia carriers. However, the response towards the screening activities was unsatisfactory as there was lack of public awareness against the importance of thalassaemia screening. An alternative approach is to screen blood donors. The purpose of this study was to observe the prevalence of thalassaemia carriers among healthy blood donors. Seven hundred and thirty eight healthy blood donors were screened in Hospital Tengku Ampuan Rahimah, Klang from July to September 2010 using cation-exchange high performance liquid chromatography (HPLC). Cases with haemoglobin variants were further analyzed by gel electrophoresis at alkaline pH. Result shows that the blood donors consisted of 413 Malays (56%), 162 Indians (22%), 148 Chinese (20%) and 15 others (2%). There were 19 (2.6%) individuals with haemoglobin E trait, six (0.8%) with co-inheritance of haemoglobin E and &alpha;&alpha;-thalassaemia and five (0.7%) with &beta;-thalassaemia trait. Haemoglobin Constant Spring and haemoglobin A2 prime were observed in two (0.3%); and Haemoglobin Lepore and alpha chain variant in one (0.2%). &alpha;&alpha;-thalassaemia and normal haemoglobin A2 &beta;-thalassaemia could not be excluded in 190 cases (26%), as they required deoxyribonucleic acid (DNA) studies for identification. Thalassaemia screening in blood donors is more feasible and effective. Therefore, a wider scale population screening including blood donors could benefit the existing thalassaemia screening programme in Malaysia.</p>\r\n', '<p>\r', '', 1417663890, 1),
(1187, 1195, 64, 'TEE BC', '', '', '', 1417652578, 0),
(1188, 1196, 64, 'AHMAD RASIDI MS', '', '', '', 1417652579, 0),
(1189, 1197, 64, 'MOHD RUSHDAN MN', '', '', '', 1417652579, 0),
(1190, 1198, 64, 'ISMAIL A', '', '', '', 1417652579, 0),
(1191, 1199, 64, 'SIDI H', '', '', '', 1417652579, 0),
(1192, 1200, 64, 'The Prevalence and Risk Factors of Sexual Dysfunction in Gynaecological Cancer Patients', '<p>\r\n	Sexual dysfunction is highly prevalent in gynaecological cancer patients. Most of the time, sexual dysfunction in gynaecological cancer is underdiagnosed as there is overlapping of symptoms with other psychological morbidities, interplaying of multiple risks, patients&rsquo; reluctance to complain or incompetence of health care provider to assess. Determining the risk factors of sexual dysfunction in cancer patients enables us to pay more attention to those who are vulnerable and to device strategies for early detection, prevention and treatment of sexual dysfunction in them. The main aim of the study was to determine the prevalence of sexual dysfunction and its risk factors in gynaecological cancer patients in Hospital Sultanah Bahiyah, Alor Star, Malaysia. Sexual function of eighty-three gynaecological cancer patients who were married were assessed with self-rated MVFSFI (Malay version Female Sexual Function Index). Self-rated WHOQOL-BREF (World Health Organization- Quality of Life- 26) which assessed the domains of quality of life was used while MINI (Mini International Neuropsychiatry Interview) was used for diagnosis of major depressive disorder. The prevalence of sexual dysfunction among the married gynaecological cancer patients was 65% (54/83). Sexual dysfunction was significantly associated with low education level (OR 3.055, CI 1.009-9.250), shorter duration of cancer (OR 0.966, CI 0.966- 0.998), ongoing chemotherapy (OR 3.045, CI 1.149-8.067), pain perception (OR 3.230, CI 1.257-8.303), absence of sexual intercourse for more than one month (OR 1.862) and three domains of quality of life such as physical health, psychological health and social relationship (OR 0.942, CI 0.908-0.978; OR 0.955, CI 0.916-0.995; OR 0.933, CI 0.894-0.973, respectively). However, sexual dysfunction was not associated with major depressive disorder (&chi;2&sup2; = 1.224, p = 0.268). The prevalence of sexual dysfunction in gynaecological cancer patients was comparable to other studies of similar population. Since, the risk factors of sexual dysfunction in gynaecological cancer patients are multidimensional, the process of assessment and management need to be holistic and patient-oriented.</p>\r\n', '<p>\r', '', 1417663903, 1),
(1193, 1201, 64, 'CHEAH YK', '', '', '', 1417656262, 0),
(1194, 1202, 64, 'The Socio-demographic Determinants of Smoking and Alcohol Consumption: A Cross Sectional Study in Penang, Malaysia', '<p>\r\n	The primary objective of the present study was to investigate the impact of socio-demographic factors on smoking and alcohol consumption among adults in Penang (Malaysia). A cross-sectional primary survey data with 398 respondents was used for analysis. The present study applied logistic regression models to examinethe factors affecting the odds of smoking and alcohol consumption. The results showed that gender, ethnicity and education could significantly affect smoking. In particular, males (OR: 26.678) had a higher likelihood of smoking compared to the females, whereas Chinese (OR: 0.177), Indians/others (OR: 0.331) and individuals with tertiary education (OR: 0.258) had a lower likelihood of smoking than others. In terms of alcohol, gender, ethnicity and marital status were found to have a significant influence on the likelihood of alcohol consumption. Specifically, males (OR: 5.051), Chinese (OR: 37.796) and Indians/others (OR: 10.863) were more likely to consume alcohol than others, while married individuals (OR: 0.380) were less likely to consume alcohol than unmarried individuals. Based on the findings of the present study, numerous population-based intervention measures were suggested.</p>\r\n', '<p>\r', '', 1417663921, 1),
(1195, 1203, 64, ' LIM JL', '', '', '', 1417657396, 0),
(1196, 1204, 64, 'AHMAD S', '', '', '', 1417657396, 0),
(1197, 1205, 64, 'A Tale of Two Boys: Case Report on Non-Consanguineous Siblings with Cerebral Palsy', '<p>\r\n	Cerebral palsy is a common cause of childhood disability. It has a great impact on parents and caregivers, especially when it reoccurs in the same family. Although familial cerebral palsy is relatively uncommon, cases have been reported among children from consanguineous, non-consanguineous marriages and multiple pregnancies suggesting a possible complex genetic mode of inheritance. Physicians need to be aware of the possibility of familial cerebral palsy for early detection and counseling. We describe a rare case of two male siblings from a non-consanguineous marriage affected by cerebral palsy.</p>\r\n', '<p>\r', '', 1417663942, 1),
(1198, 1206, 64, 'SUNDARESAN AN', '', '', '', 1417657926, 0),
(1199, 1207, 64, 'VIKRAM M', '', '', '', 1417657926, 0),
(1200, 1208, 64, 'MOHANDAS K', '', '', '', 1417657926, 0),
(1201, 1209, 64, 'NUR FARHANA MY', '', '', '', 1417657926, 0),
(1202, 1210, 64, 'POTTURI GOWRI S', '', '', '', 1417657927, 0),
(1203, 1211, 64, 'Impact of Total Contact Plaster Boot via off Loading Practice in Trophic Ulcers of the Feet: Case Series', '<div>\r\n	Trophic ulcers have emerged as one of the major complications following diabetes mellitus (DM) and Hansen&rsquo;s diseases (HD). In this case series, the study attempted total contact plaster boot using a readily available plaster of Paris to treat trophic ulcer for 10 subjects. A total of five subjects with DM and five subjects with HD were included based on the study criteria. Pre and post test measure of wound measurement size following total contact plaster boot were taken as an outcome measure. All ten subjects showed decrease in size of wound following fifteen days of treatment. No adverse effects were associated with this type of treatment. Subjects with trophic ulcer may benefit from the application of total contact plaster boot.</div>\r\n', '<div>\r', '', 1417664047, 1),
(1204, 1212, 64, 'REYNU R', '', '', '', 1417660094, 0),
(1205, 1213, 64, 'AZNAN M', '', '', '', 1417660094, 0),
(1206, 1214, 64, 'TAHER MM', '', '', '', 1417660094, 0),
(1207, 1215, 64, 'Uncommon Presentation of a Common Aesthetic Procedure: Late Infection of a Ruptured Silicone Breast Implant', '<p>\r\n	Breast augmentation using silicone implant is a common and popular aesthetic procedure with a high safety profile. Infection of silicone breast implants is a rare clinical entity, with skin commensal organisms often implicated as causative pathogen. Surgical treatment often involves removal of the implant with salvage procedures limited to selected cases. This case highlights a delayed presentation of an infected silicone breast implant presenting as a chest wall abscess 15 years after initial surgery.</p>\r\n', '<p>\r\n	Breast augmentation using silicone implant is a common and popular aesthetic procedure with a high safety profile. Infection of silicone breast implants is a rare clinical entity, with skin commensal organisms often implicated as causative pathogen. Surgical treatment often involves removal of the implant with salvage procedures limited to selected cases. This case highlights a delayed presentation of an infected silicone breast implant presenting as a chest wall abscess 15 years after initial surgery.</p>\r\n', '', 1417664114, 1),
(1208, 1216, 97, 'Wan Ahmad L', '', '', '', 1419043928, 1),
(1209, 1217, 97, 'Tan ZY', '', '', '', 1419043994, 1),
(1210, 1218, 97, 'Nur Sharifah MS', '', '', '', 1419044060, 1),
(1211, 1219, 97, 'Jaafar MZ', '', '', '', 1419044111, 1),
(1212, 1220, 97, 'Sharaf I', '', '', '', 1419044138, 1),
(1213, 1221, 64, 'The Impact Of A Patient Education Package On Outcomes Of Pain Management After Orthopaedic Surgery In A Tertiary Hospital, Malaysia', '<p>\r\n	Patient&rsquo;s belief towards pain management may affect pain management outcomes and quality of life. The purpose of this study was to determine the impact of a preoperative pain education package towards pain belief among patients undergoing orthopaedic surgery in a tertiary hospital. A one-group pre-test post-test design study was conducted on orthopaedic surgery patients. Thirty respondents were recruited and preoperative pain education was administered individually before surgery. Preoperative and postoperative pain belief, management scores and side effects were measured using the Barrier Questionnaire (BQ-13). The results reported significant differences between pre-test scores (Mean=41.87, Standard Deviation=11.467) and post-test scores (Mean=34.80, Standard Deviation=13.026) of pain belief (t = 2.84, p = 0.004). There were also significant differences between pre-test scores (Mean=37.10, Standard Deviation=10.610) and post-test scores (Mean=30.80, Standard Deviation=11.424) of pain management (t= 3.856, p = 0.0005). Respondent&rsquo;s gender (t = -2.403, p = 0.023) and ethnicity (F = 5.038, p=0.014) reported significant differences with p value &lt; 0.05 respectively. However, there were no significant differences between educational level, ethnicity, prior surgical history with pain belief (p &gt; 0.05). There was positive impact of the pain education package towards pain belief and pain management among respondents who underwent orthopaedics surgery in a tertiary hospital. Reinforcement of pain educational program is pivotal in order to achieve optimal postoperative pain management.</p>\r\n', '<p>\r', '', 1421120653, 1),
(1214, 1222, 456, 'Mardiana binti Mansor', '', '', '', 1419386351, 1),
(1215, 1223, 456, 'Ass Prof Dr San San Oo', '', '', '', 1419386463, 1),
(1216, 1224, 456, 'Ass Prof Dr. Khatijah Lim Abdullah', '', '', '', 1419386622, 1),
(1217, 1225, 456, 'Associate Professor Dr. San San Oo', '', '', '', 1419386687, 1),
(1218, 1226, 64, 'Vol. 9 No. 2 : 2014', '', '', '', 1419924141, 1),
(1219, 1227, 64, 'The History of RNAi and MicroRNA Discovery', '<p>\r\n	-</p>\r\n', '<p>\r\n	-</p>\r\n', '', 1419923654, 1),
(1220, 1228, 64, 'Pharmacovigilance and Reporting Adverse Drug Reactions', '<p>\r\n	-</p>\r\n', '<p>\r\n	-</p>\r\n', '', 1419923671, 1),
(1221, 1229, 64, 'AMEET KJ', '', '', '', 1419911279, 0),
(1222, 1230, 64, 'MURLIMANJU BV', '', '', '', 1419911279, 0),
(1223, 1231, 64, 'A Morphometric Analysis of Intercondylar Notch of Femur with Emphasis on Its Clinical Implications', '<p>\r\n	The intercondylar notch has been an anatomic site of interest as it lodges the anterior cruciate ligament. The objectives of the present study were to study the&nbsp;morphology and morphometry of femoral intercondylar notch in cadaveric dry bones with emphasis on its clinical implications.The present investigation was performed by using 97 dry femora. The parameters like intercondylar notch width, intercondylar notch depth, condylar width and condylar depth were measured. The shapes of intercondylar notch were also analyzed. The measurements were compared statistically with respect to right and sides and were tabulated. It was observed that the intercondylar notch was having inverted &lsquo;U&rsquo; shape morphology in 71 (73.2%) specimens and it was inverted &lsquo;V&rsquo; shaped in 26 (26.8%) cases. The mean intercondylar notch width, intercondylar notch depth, condylar width and condylar depth were 11.9 &plusmn; 2.7 mm, 26.3 &plusmn; 2.4 mm, 72.9 &plusmn; 5.3 mm and 57.3 &plusmn; 4.3 mm, respectively. It was observed that there was no statistical significance difference observed (p &gt; 0.05) between the right and left sides. The notch width index and notch depth index were determined as 0.25 and 0.46, respectively. The morphometry data of the present study could provide importance to the orthopedicians in prevention and management of knee injuries. We believe that the present study has provided additional information on this subject and these data might be of use to the clinicians who are involved in the diagnosis and management of knee problems.</p>\r\n', '<p>\r', '', 1419923687, 1),
(1224, 1232, 64, 'VENGATASUBRAMANI M', '', '', '', 1419911731, 0),
(1225, 1233, 64, ' VIKRAM M', '', '', '', 1419911731, 0),
(1226, 1234, 64, 'Physical Training Improved Cardiovascular Fitness Level among Chronic Obstructive Pulmonary Disease Patients', '<p>\r\n	Chronic obstructive pulmonary disease (COPD) patients are reported to have cardiovascular instability which leads to greater limitation for activities there by leads to poor quality of life. Physical training proved to be one of the moderators of these limitations. However, uncertainty prevails among the protocol and duration. The present study investigated the effect of physical training on blood pressure,&nbsp;heart rate and Rate pressure Product (RPP) among COPD patients. A total of thirty COPD patients aged between 40 to 55 years were recruited for the study based on the inclusion and exclusion criteria and were assigned in to experimental group (15 patients) and control group (15 patients). The mean difference of blood pressure, heart rate and RPP were analyzed using paired t-test. There was significant difference between the pre and post test values of all parameters between experimental and control group with p&lt;0.05. Diastolic blood pressure showed to be less significant compared to the systolic blood pressure. This showed that specifically designed physical improved the cardiovascular fitness among COPD patients.</p>\r\n', '<p>\r', '', 1419923704, 1),
(1227, 1235, 64, 'AIDA J', '', '', '', 1419912259, 0),
(1228, 1236, 64, 'SITI FATIMAH S', '', '', '', 1419912259, 0),
(1229, 1237, 64, 'MOHD MUHAIMIN A', '', '', '', 1419912259, 0),
(1230, 1238, 64, 'CHAI SY', '', '', '', 1419912259, 0),
(1231, 1239, 64, 'SAHRINA W', '', '', '', 1419912259, 0),
(1232, 1240, 64, 'TEO BH', '', '', '', 1419912260, 0),
(1233, 1241, 64, 'Psychological Disorders and Help Seeking Behaviour among Malaysian Medical Students in Their Clinical Years', '<div>\r\n	The present study is to determine the level of depression, anxiety and stress among Universiti Kebangsaan Malaysia (UKM) clinical year medical students and their help seeking behaviour in relation to their psychological aspect. A cross-sectional study was conducted in UKM Medical Centre for three months duration. The study population was the third, fourth and fifth year medical students via proportional sampling. They were given questionnaires consisting of socio-demographic items, Depression, Anxiety and Stress Scale 21 &ndash; Bahasa Malaysia version BM-DASS21 and questions regarding help-seeking behaviors. Psychometric assessment (BM-DASS21) estimated the level of depression, anxiety and stress among medical students. Help-seeking behavior questions assessed the use of medical facilities and other help-seeking sources. Regarding questions on the barriers towards seeking help from the primary health-care, three parts were evaluated: structural barriers, barriers towards perceptions on mental-health disorder and barriers towards use of primary health-care services. There were 380 medical students who were involved in this research. It was found that some students experienced depression (1.3%), anxiety (2.4%) and stress (2.4%). Majority of them preferred to seek help from friends (283; 74.5%) followed by parents (275; 72.4%). There was a significant association between students who were anxious and help seeking behaviour from formal sources: counselor (p=0.001) and religious personnel (p=0.03). Hence, it is essential to increase their awareness on psychological disorders and more efficient screening programme should be used to recognize medical students with possible emotional disorders.</div>\r\n', '<div>\r', '', 1419923723, 1),
(1234, 1242, 64, 'TAN TL', '', '', '', 1419914935, 0),
(1235, 1243, 64, 'NOOR ASMIDAR A', '', '', '', 1419914936, 0),
(1236, 1244, 64, 'ONG WJ', '', '', '', 1419914936, 0),
(1237, 1245, 64, 'AHMAD FUAD FAHMI MN', '', '', '', 1419914936, 0),
(1238, 1246, 64, 'CHIENG ZL', '', '', '', 1419914936, 0),
(1239, 1247, 64, ' AKMAL SI', '', '', '', 1419914936, 0),
(1240, 1248, 64, 'The 28-Day Mortality Prediction in Sepsis Patients Using Static Lactate Concentration and Early Lactate Clearance: An Observational Study', '<p>\r\n	Sepsis causes high mortality and morbidity. Static lactate concentration and early lactate clearance are cited to be a predictor for sepsis survival. This study examined the clinical utility of static lactate concentration and early lactate clearance within the first six hours of admission in Emergency Department (ED) to predict 28-day mortality rate in sepsis patients. Patients who presented with sepsis, severe sepsis or septic shock and admitted to ED of Universiti Kebangsaan Malaysia Medical Centre were recruited. Blood lactate concentrations were measured upon admission (H0), at 1st hour (H1) and 6th hour (H6), respectively. Either standard treatment of sepsis or early goal directed therapy was initiated according to sepsis severity. A follow-up report was conducted at 28 days via telephone call, e-mail or case notes. Patients were later classified into survivor and non-survivor as final outcome. Static lactate concentration appeared to be significantly higher for non-survivor as compared to the survival group at H0, H1 and H6 (p&lt;0.05). The lactate clearance trend reflects no relationship between early lactate clearance and 28-day mortality. Static lactate concentration showed a superior predictor for sepsis over early lactate clearance. Although early lactate clearance was unable to prove its ability to predict 28-day mortality, our findings suggest it can be a useful tool to gauge the resuscitation outcome.</p>\r\n', '<p>\r', '', 1419923884, 1),
(1241, 1249, 64, 'ROKIAH O', '', '', '', 1419919917, 0),
(1242, 1250, 64, 'UMI KMN', '', '', '', 1419919917, 0),
(1243, 1251, 64, ' LEOW SN', '', '', '', 1419919917, 0),
(1244, 1252, 64, 'Long Term Outcome in Unilaterally Treated Retinopathy of Prematurity: A Case Report', '<p>\r\n	Retinopathy of prematurity (ROP) is a disorder describing an immature vascularisation of a developing retina in low birth weight preterm infants. This condition potentially leads to blindness. ROP developed as a response of hypoxia of the eye due to incomplete development of the retinal vessels. ROP is commonly reported as bilateral disease,a small percentage of infants have asymmetrical changes. We report a case of long-term outcome of a asymmetry ROP changes with peripheral retinal ablation in a single eye. This particular case demonstrates the possible long-term outcome of unilaterally treated ROP which could either be due to the severity of the disease itself or the treatment she received. It is important to highlight the possibility of unequal development of the eye in asymmetrical presentation of ROP.&nbsp;</p>\r\n', '<p>\r', '', 1419923941, 1),
(1245, 1253, 64, 'AHMAD NABIL MR', '', '', '', 1419920496, 0),
(1246, 1254, 64, ' AZLIN B', '', '', '', 1419920496, 0),
(1247, 1255, 64, 'Managing ‘Schizophrenia in the Midst of Schizophrenia’- The Role of Assertive Community Treatment', '<p>\r\n	This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with two other family members with the same illness and their carer who developed major depression from shouldering the burden of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.</p>\r\n', '<p>\r\n	This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with two other family members with the same illness and their carer who developed major depression from shouldering the burden of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.</p>\r\n', '', 1419920497, 1),
(1248, 1256, 64, 'MARHANI M', '', '', '', 1419920747, 0),
(1249, 1257, 64, 'AZLIN B', '', '', '', 1419920747, 0),
(1250, 1258, 64, 'Managing ‘Schizophrenia in the Midst of Schizophrenia’- The Role of Assertive Community Treatment', '<p>\r\n	This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with two other family members with the same illness and their carer who developed major depression from shouldering the burden of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.</p>\r\n', '<p>\r\n	This case report emphasizes the role of Assertive Community Treatment (ACT) in managing a family with four members suffering from mental illness, mainly schizophrenia. We report a case of middle-aged lady who was diagnosed with treatment resistant schizophrenia (TRS) living with two other family members with the same illness and their carer who developed major depression from shouldering the burden of caring for mentally ill family members. ACT, through its holistic approach, proved to have reduced hospitalizations and improve symptom control and quality of life in this family.</p>\r\n', '', 1419923628, 1),
(1251, 1259, 64, 'ABDIKARIM M', '', '', '', 1419922883, 0),
(1252, 1260, 64, 'JASMAN MH', '', '', '', 1419922883, 0),
(1253, 1261, 64, ' TAHER MM', '', '', '', 1419922883, 0),
(1254, 1262, 64, 'IDRIS MA', '', '', '', 1419922883, 0),
(1255, 1263, 64, 'HARUNARASHID H', '', '', '', 1419922883, 0),
(1256, 1264, 64, 'Multiple Acute Aortic Dissection in a Young Adult', '<p>\r\n	The diagnosis of aortic dissection in a young adult in the absence of atherosclerosis or prior history of trauma is extremely rare. The presence of more than one arterial dissection site in such a patient is even more unheard of. We highlight a case of spontaneous multiple acute arterial dissections occurring in a 32-year-old male. Stanford B aortic dissection and a separate dissection extending from the bifurcation of the right common iliac artery to the right common femoral artery was noted on computed tomographic angiography (CTA). A small aneurysm of the right subclavian artery was also noted. A two-stage hybrid procedure involving a combination of open and endovascular surgery was employed. The rarity and lethality of this condition warrants a high index of suspicion for early diagnosis and prompt intervention.</p>\r\n', '<p>\r', '', 1419923060, 1),
(1257, 1265, 64, 'PREMALATHA S', '', '', '', 1419923310, 0),
(1258, 1266, 64, 'MARYAM MF', '', '', '', 1419923311, 0),
(1259, 1267, 64, 'Orogastric Lavage: A Lifesaving Procedure Now Forgotten', '<p>\r\n	Orogastric lavage has been performed since 200 years ago for intoxicated patients. Due to the risk that outweighs benefits it has fallen out of favour for the last decade. A teenage girl presented to Emergency Department with history of ingestion of a bottle of pesticide within the time frame before gastric emptying. The girl was resuscitated, intubated and orogastric lavage was performed. Fifty cc of the toxic substance was siphoned and antidote of the toxin was administered. She was admitted to the Intensive Care Unit, subsequently recovered and discharge five days later. Definitive airway management, proper technique, correct selection of patients and adequate monitoring are paramount to the success of orogastric lavage.</p>\r\n', '<p>\r', '', 1419923994, 1),
(1260, 1268, 470, 'Kirubakaran Krishnamoorthy Gunasekaran', '', '', '', 1420011899, 1),
(1261, 1269, 470, 'Effect of Wobble Board Balance Training Program Among Athletes with Functionally Unstable Ankle Joint', 'The objective of the study was to find out the effect of wobble board training program on balance in athletes with Functional Ankle Instability (FAI). This study determines the effect of wobble board training on balance in athlete with functional ankle instability (FAI). A total of 30 subjects (n=30) with FAI were recruited based on the set inclusion and exclusion criteria. All the subjects were evaluated for balance using single limb stance test and functional reach test and they were subjected to wobble board training program for 5 sessions in a week for 4 weeks. Independent sample t test was used to compare the mean between the pre and post test data’s and the level of significance was set at level of 0.05. In single limb stance test, the athletes scored high post training score mean value [26.4] than pre training score [9.73].Thus the difference is statistically confirmed by the obtained‘t’ value [16.00]. In functional reach test, the athletes scored high post training score mean value [15.88] than pre training score (14.26). Thus the difference was statistically confirmed by the obtained‘t’ value [12.54]. This indicated that wobble board training promoted the static and dynamic balance in athletes with functionally unstable ankle. Hence, it can be concluded that wobble board training is effective in improving balance among athletes with functionally unstable ankle                       ', 'The objective of the study was to find out the effect of wobble board training program on balance in athletes with Functional Ankle Instability (FAI). This study determines the effect of wobble board training on balance in athlete with functional ankle instability (FAI). A total of 30 subjects (n=30) with FAI were recruited based on the set inclusion and exclusion criteria. All the subjects were evaluated for balance using single limb stance test and functional reach test and they were subjected to wobble board training program for 5 sessions in a week for 4 weeks.', '', 1420012480, 1),
(1262, 1270, 64, 'KUMAR R', '', '', '', 1420015327, 0),
(1263, 1271, 64, ' FARIZAL F', '', '', '', 1420015328, 0),
(1264, 1272, 64, 'SHUHAILI MA', '', '', '', 1420015328, 0),
(1265, 1273, 64, ' RAZMAN J', '', '', '', 1420015328, 0),
(1266, 1274, 64, 'SHAHIZON AMM', '', '', '', 1420015328, 0),
(1267, 1275, 64, 'Vein of Galen Malformation', '<p>\r\n	-</p>\r\n', '<p>\r\n	-</p>\r\n', '', 1420015536, 1),
(1268, 1276, 473, 'Norshamsiah Md Din', '', '', '', 1420511741, 1),
(1269, 1277, 473, 'Wan Haslina Wan Abdul Halim', '', '', '', 1420518451, 1),
(1270, 1278, 473, 'Fuad Ismail', '', '', '', 1420518607, 1),
(1271, 1279, 64, 'FLOURESCEIN ANGIOGRAM AND FUNDUSCOPIC FEATURES OF RADIATION INDUCED RETINOPATHY: A DESCRIPTIVE VALUE OF RETINOPATHY SEVERITY IN PATIENTS WITH NASOPHARYNGEAL CARCINOMA ', '<p>\r\n	Radiation retinopathy (RR) is a known complication after radiotherapy for Nasopharyngeal Carcinoma (NPC). This study aims to relate the relationship of RR and radiation dose in patients with NPC through assessment with clinical funduscopy and fundus fluorescein angiogram (FFA). A cross sectional study was conducted on patients with NPC who had completed radiotherapy treatment in the Oncology Clinic, University Kebangsaan Malaysia Medical Centre (UKMMC). 82 eyes of 42 patients were examined and the prevalence of RR was found to be 35.4%. The severity of RR is strongly associated with the dose of radiation to the retina (Spearman correlation value=0.48; p&lt;0.001). The common features of RR assessed by FFA were telangiectatic vessels (26.2%) and capillary non-perfusion (14.3%). Retinal neovasularization occurred in 10.7% of eyes. The level of visual deterioration correlated with the severity of RR with 26% of eyes experiencing a visual acuity of 6/18 or worse. More than one third of patients developed RR, with radiation maculopathy being the commonest cause for significant visual loss. FFA is a useful tool in detecting early signs of radiation retinopathy and maculopathy.</p>\r\n', '<p>\r', '', 1422259219, 1),
(1272, 1280, 484, 'Anamika Kumari', '', '', '', 1420921226, 1),
(1273, 1281, 484, 'Abhishek Prasad Sinha', '', '', '', 1420921288, 1),
(1274, 1282, 484, 'Anuj Jain', '', '', '', 1420921334, 1),
(1275, 1283, 484, 'B.V. Murlimanju', '', '', '', 1420921397, 1),
(1276, 1284, 484, 'Divya Premchandran', '', '', '', 1420921434, 1),
(1277, 1285, 484, 'Prashanth K.U.', '', '', '', 1420921491, 1),
(1278, 1286, 484, 'Latha V. Prabhu', '', '', '', 1420921565, 1),
(1279, 1287, 484, 'Vasudha V. Saralaya', '', '', '', 1420921625, 1),
(1280, 1288, 484, 'Teresa Joy', '', '', '', 1420921675, 1),
(1281, 1289, 484, 'Ashwin R. Rai', '', '', '', 1420921721, 1),
(1282, 1290, 64, 'Morphology of nutrient foramina in human metatarsals and their clinical importance', '<p>\r\n	The objectives were to study the morphology of the nutrient foramina in metatarsals and to figure out their foramina index. The present study included 271 unpaired, human metatarsals. The age and sex of the metatarsals were not known. Nutrient foramina of each bone were grossly examined with respect to their topography and number. The indexing of the foramina was performed with the formula of Hughes. The morphometry of the present study was performed with the application of digital Vernier caliper. It was observed that, 79.3% of metatarsals had single nutrient foramen, 9.2% had double foramina, 1.1% exhibited three foramina and in 10.4% of metatarsals, there was no foramen. The mean index of the foramina of 1st metatarsal, 2nd metatarsal, 3rd metatarsal, 4th metatarsal and 5th metatarsal were 65.3, 43.6, 44.4, 45.3 and 47.5 respectively. It was observed that most of the metatarsals had single nutrient foramen. The locations of the nutrient foramen varied from 1st to 5th metatarsals. In the 1st metatarsal and 2nd metatarsals, the foramina were observed at their lateral side and in the rest; they were commonly observed at the medial surface. The morphological distribution about nutrient foramina is of very much required in some surgeries like utilization of metatarsal bones in the foot reconstruction. The morphological data is also enlightening to the plastic surgery in case of bone graft microvascular operations.</p>\r\n', '<p>\r', '', 1422258898, 1),
(1283, 1291, 266, 'Dazlin Masdiana Sabardin', '', '', '', 1421197369, 1),
(1284, 1292, 266, 'Colin Robertson', '', '', '', 1421197544, 1),
(1285, 1293, 64, '"Focal" Cardiac Concussion – an under-recognized problem?', '<p>\r\n	Cardiac concussion is a sudden, direct blunt trauma cause to the chest which led to death. However, there are patients who did not completely fulfil this definition. We report two cases which did not fit into the definition domain. Two male patients presented to emergency department with moderate anterior chest pain after motor vehicle crash showed transient anterior ST segment elevation at chest lead V2 and V3 with raised creatine kinase and normal troponin T. The electrocardiogram changes fully resolved after 24 hours. Both patients were discharged uneventfully after 24 hours monitoring in emergency department short-stay ward. Conventional definition of cardiac concussion (commotion cordis) and cardiac contusion may be unsuitable to describe these cases. Therefore, we propose the diagnosis of focal cardiac concussion. We also highlighted the ability of emergency department to manage these patients in short-stay ward.</p>\r\n', '<p>\r', '', 1422319852, 1),
(1286, 1294, 207, 'Review of Morphology of nutrient foramina in human metatarsals and their clinical importance', '', '', '', 1422263978, 0),
(1287, 1295, 507, 'Norfazilah A', '', '', '', 1422422013, 1),
(1288, 1296, 507, 'Hafizah Z', '', '', '', 1422424049, 1),
(1289, 1297, 507, 'Siti Zubaidah S', '', '', '', 1422424128, 1),
(1290, 1298, 507, 'Azmawati MN', '', '', '', 1422424176, 1);
INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
(1291, 1299, 64, 'Poor Peer Support as a Predictive Factor towards Depression among Adolescent in a state of Selangor, Malaysia', '<p>\r\n	Lately symptoms of depression among adolescents have been alarmingly increasing. This study was conducted to determine the prevalence of depression and its predictive factors. A cross-sectional study was conducted on randomly selected 191 adolescent consisting of Form Four students from five selected secondary schools in the state of Selangor, Malaysia. A self-administrative questionnaire which consists of six sections (A) demographics, (B) level of depression, (C) family relationships, (D) peer support, (E) level of self-esteem, and (F) academic achievement were handed out. The prevalence of depression was 50.3%. Multiple logistic regression analysis found that adolescents who had problems with peers were more likely to suffer from depression compared to those without problems with their peers (aOR 2.84, 95% CI 1.50, 5.36). Future researches need to examine additional factors such as pressure from the teachers in order to strengthen our understanding about depression among adolescents. It is hoped that these findings can be useful to various parties who are concerned about this problem.</p>\r\n', '<p>\r', '', 1423036036, 1),
(1292, 1300, 275, 'SUARN S', '', '', '', 1422759435, 1),
(1293, 1301, 64, 'A case report of a patient involved in an alleged homicide–attempted suicide case - A theoretical discussion on the theory of homicide-suicide.', '<p>\r\n	This case report stresses the role of depression in the manifestation of a homicide-attempted suicide case. We report a case of a man who was alleged of murder his partner and attempted suicide. Previously he had several failures and rejections in relationships on top of work-related stress. He was diagnosed with Major depressive disorder and treated with antidepressant. The manifestation of homicide-suicide will be discussed from various theoretical points of view.</p>\r\n', '<p>\r\n	This case report stresses the role of depression in the manifestation of a homicide-attempted suicide case. We report a case of a man who was alleged of murder his partner and attempted suicide. Previously he had several failures and rejections in relationships on top of work-related stress. He was diagnosed with Major depressive disorder and treated with antidepressant. The manifestation of homicide-suicide will be discussed from various theoretical points of view.</p>\r\n', '', 1423034972, 1),
(1294, 1302, 589, 'NURUL IZZAH', '', '', '', 1426739953, 1),
(1295, 1303, 589, 'ROSMILAH MISNAN', '', '', '', 1426740062, 1),
(1296, 1304, 589, 'ZAILATUL HANI MOHAMAD YADZIR', '', '', '', 1426750243, 1),
(1297, 1305, 589, 'FAIZAL BAKHTIAR', '', '', '', 1426750332, 1),
(1298, 1306, 589, 'NOORMALIN ABDULLAH', '', '', '', 1426750377, 1),
(1299, 1307, 589, 'SHAHNAZ MURAD', '', '', '', 1426750421, 1),
(1300, 1308, 64, 'IDENTIFICATION OF MAJOR AND MINOR ALLERGENS OF MUD CRAB (SCYLLA SERRATA)', '<p>\r\n	Crab meat is a valuable nutrient source of proteins and functional lipids and is widely consumed. However the prevalence of crab allergy has increased over the past few years. In order to better understand crab allergy, it is necessary to identify crab allergens. The aim of this study was to compare the IgE-binding proteins of raw and cooked extracts of mud crab (Scylla serrata). Raw and cooked extracts of the mud crab were prepared. Protein profiles and IgE reactivity patterns were identified by sodium dodecyl sulfate polyacrylamide gel electrophoresis (SDS-PAGE) followed by immunoblotting using sera from 21 skin prick test (SPT) positive patients. In SDS-PAGE, 20 protein bands (12 to 250 kDa) were observed in the raw extract while the cooked extract demonstrated fewer bands. Protein bands between 40 to 250 kDa were sensitive to heat denaturation and no longer observed in the cooked extract. In immunoblotting experiments, raw and cooked extracts demonstrated 11 and 4 IgE-binding proteins, respectively, with molecular weights of between 23 and 250 kDa. A heat-resistant 36 kDa protein, corresponding to crab tropomyosin was identified as the major allergen of both extracts. In addition, a 41 kDa heat-sensitive protein believed to be arginine kinase was shown to be a major allergen of the raw extract. Other minor allergens were also observed at various molecular weights.</p>\r\n', '<p>\r', '', 1435033214, 1),
(1293, 1309, 275, 'A case report of a patient involved in an alleged homicide–attempted suicide case - A theoretical discussion on the theory of homicide-suicide.', 'This case report stresses the role of depression in the manifestation of a homicide-attempted suicide case. We report a case of a man who was allegedly murder his partner and then attempted suicide. Previously he had several failures and rejections in relationships on top of work-related stress. He was diagnosed with major depressive disorder and treated with antidepressant. The scarcity of homicide-attempted suicide as exemplified in this case could give an insight to the psychiatrist on how it could be understood and possibly be prevented.\r\n', 'This case report stresses the role of depression in the manifestation of a homicide-attempted suicide case. We report a case of a man who was allegedly murder his partner and then attempted suicide. Previously he had several failures and rejections in relationships on top of work-related stress. He was diagnosed with major depressive disorder and treated with antidepressant. The scarcity of homicide-attempted suicide as exemplified in this case could give an insight to the psychiatrist on how it could be understood and possibly be prevented.\r\n', 'I have uploaded a few edited files. Please refer to cover letter. edition made, marked in RED.', 1429136881, 1),
(1301, 1310, 64, 'Vol. 10 No. 1 : 2015', '', '', '', 1432888646, 1),
(1302, 1311, 64, 'TAN HC', '', '', '', 1432801872, 0),
(1303, 1312, 64, 'PUAH JWD', '', '', '', 1432801873, 0),
(1304, 1313, 64, 'APIPI M', '', '', '', 1432801873, 0),
(1305, 1314, 64, 'SOHAMI AE', '', '', '', 1432801874, 0),
(1306, 1315, 64, 'MAHAT NF', '', '', '', 1432801874, 0),
(1307, 1316, 64, 'Acne Disability, Self Management and Help-Seeking Behaviour among Medical Students', '<p>\r\n	Acne can affect personal appearance and impair quality of life. A cross sectional study, over a period of 12 months was conducted among medical students at Universiti Kebangsaan Malaysia to evaluate the disability, self management and help-seeking behaviour of medical students for acne. A total of 361 students were selected using stratified cluster random sampling. Acne was graded using the Comprehensive Acne Severity Score (CASS) while acne disability was assessed using the Cardiff Acne Disability Index (CADI). Acne self management and help-seeking behaviour was assessed using the acne management questionnaire. Mean CADI score was 3.35 + 2.39, with mostly mild (83.8%, n=206) and moderate (14.6%, n=36) levels of impairment. Female students and those from the non-Malay ethnic group demonstrated higher disability (3.76 + 2.24, p=0.006 and 3.79 + 2.59, p=0.018, respectively). Although the majority of students (87.4%, n=215) knew that acne can be treated, they preferred to discuss their acne problems with friends (54.1%, n=133) rather than consulting a physician (9.3%, n=23). Most of them also practiced non-evidence based measures for their acne. The results of the present study shows that young adults tend to have mild grades of acne severity and disability. However, efforts are needed to enhance their knowledge on evidence based management of acne and modify their help seeking behavior. This would help reduce complications such as permanent scars and improve quality of life, as acne is a treatable dermatological disorder.</p>\r\n', '<p>\r', '', 1432888397, 1),
(1308, 1317, 64, 'AlFatih P', '', '', '', 1432857469, 0),
(1309, 1318, 64, 'Munirah MI', '', '', '', 1432857470, 0),
(1310, 1319, 64, 'Ainaen M', '', '', '', 1432857470, 0),
(1311, 1320, 64, 'Wenda L', '', '', '', 1432857471, 0),
(1312, 1321, 64, 'Sharifa Ezzat WP', '', '', '', 1432857471, 0),
(1313, 1322, 64, 'Comparison of Immunohistochemical Markers in Core Needle Biopsy and Excisional Biopsy in Breast Carcinoma', '<div>\r\n	Estrogen receptor (ER), progesterone receptor (PR) and human epidermal growth factor receptor 2 (HER2) immunohistochemistry are important markers in the management of patient with breast carcinoma. In this study, we determine the concordance rate of ER, PR and HER2 immunohistochemistry markers between core needle biopsy (CNB) and excisional biopsy (EB) of breast carcinoma in patients of Universiti Kebangsaan Malaysia Medical Centre (UKMMC) from January 2002 until December 2012. A total of 93 female patients with CNB and subsequent EB were included in this retrospective descriptive study. Immunohistochemistry is used to determine ER, PR and HER2. ER and PR was graded using Allred score (0 to 8) while HER2 was scored from 0 to 3+. The markers between these two biopsies were compared to determine the concordance rate. In ER and PR, 93 samples were compared. ER was concordant in 80 cases (86.0%) and 13 cases (14.0%) was discordant. PR was concordant in 82 cases (88.2%) and discordant in 11 cases (11.8%). In HER2, 87 samples were compared and 62 cases (71.3%) were concordant while 25 cases (28.7%) were discordant. Concordance between CNB and EB was high for ER and PR. However, concordance rate for HER2 immunohistochemistry was less consistent. Overall, immunohistochemical analyses of CNB reflect the tumour marker status of the excised specimen.</div>\r\n', '<div>\r', '', 1432888427, 1),
(1314, 1323, 64, 'Sundaraganesh K', '', '', '', 1432857789, 0),
(1315, 1324, 64, ' Mohandas K', '', '', '', 1432857790, 0),
(1316, 1325, 64, 'Kirubakaran KG', '', '', '', 1432857790, 0),
(1317, 1326, 64, 'Effect of Wobble Board Balance Training Program among Athletes with Functionally Unstable Ankle Joint', '<p>\r\n	The objective of the study was to find out the effect of wobble board training program on balance in athletes with Functional Ankle Instability (FAI). &nbsp;A total of 30 subjects (n=30) with FAI were recruited based on the set inclusion and exclusion criteria. All the subjects were evaluated for balance using single limb stance test and functional reach test and they were subjected to wobble board training program for five sessions in a week for four weeks. Paired sample t test was used to compare the mean between the pre and post test data&rsquo;s and the level of significance was set at level of 0.05. In single limb stance test, the athletes scored high post training score mean value [26.4] than pre training score [9.73]. Thus the difference is statistically confirmed by the obtained&lsquo;t&rsquo; value [16.00]. In functional reach test, the athletes scored high post training score mean value [15.88] than pre training score (14.26). Thus the difference was statistically confirmed by the obtained&lsquo;t&rsquo; value [12.54]. This indicated that wobble board training promoted the static and dynamic balance in athletes with functionally unstable ankle. Hence, it can be concluded that wobble board training might be effective in improving balance among athletes with functionally unstable ankle. &nbsp;</p>\r\n', '<p>\r', '', 1432888453, 1),
(1318, 1327, 64, 'Norshamsiah MD', '', '', '', 1432874262, 0),
(1319, 1328, 64, 'Kok HS', '', '', '', 1432874263, 0),
(1320, 1329, 64, 'Flourescein Angiogram and Funduscopic Features of Radiation Induced Retinopathy: A Descriptive Value of Retinopathy Severity in Patients with Nasopharyngeal Carcinoma ', '<div>\r\n	Radiation retinopathy (RR) is a known complication after radiotherapy for Nasopharyngeal Carcinoma (NPC). This study aims to relate the relationship of RR and radiation dose in patients with NPC through assessment with clinical funduscopy and fundus fluorescein angiogram (FFA). A cross sectional study was conducted on patients with NPC who had completed radiotherapy treatment in the Oncology Clinic, Universiti Kebangsaan Malaysia Medical Centre (UKMMC). Eighty two eyes of 42 patients were examined and the prevalence of RR was found to be 35.4%. The severity of RR is strongly associated with the dose of radiation to the retina (Spearman correlation value=0.48; p&lt;0.001). The common features of RR assessed by FFA were telangiectatic vessels (26.2%) and capillary non-perfusion (14.3%). Retinal neovasularization occurred in 10.7% of eyes. The level of visual deterioration correlated with the severity of RR with 26% of eyes experiencing a visual acuity of 6/18 or worse. More than one third of patients developed RR, with radiation maculopathy being the commonest cause for significant visual loss. FFA is a useful tool in detecting early signs of radiation retinopathy and maculopathy.</div>\r\n', '<div>\r', '', 1432888471, 1),
(1321, 1330, 64, 'Leong JWS', '', '', '', 1432881923, 0),
(1322, 1331, 64, 'Lye MS', '', '', '', 1432881923, 0),
(1323, 1332, 64, 'Functional Status of Post Stroke Survivors ', '<div>\r\n	Globally, stroke is the commonest cause of long-term disability. The residual disabilities among post stroke patients affect their daily living activities. The aim of rehabilitation therapy is to help stroke survivors to gain back their functional ability. The present study aimed to determine the relationship between post stroke duration with functionality status of post-stroke survivals at a teaching hospital in Kuala Lumpur. A cross-sectional study involved one hundred nine five post stroke patients who attended the Rehabilitation Clinic over a 4-month period. The data on &nbsp;post stroke duration was reveale from patient&rsquo;s cleckship. Their functional status was assessed with Modified Barthel Index (MBI). Results showed the mean age of participants was 61 years (SD=13.86, range:22-87 years), with 118 males and 81 females &nbsp;having &nbsp;a median duration of 12 months post-stroke (range: 1&ndash;79 months). The prevalence 123 (63.1%) of stroke survivors are found to be dependent in their daily living activity. A Chi-square test for independence &nbsp;indicated there was significant relationship between post-stroke duration with levels of functioning, &chi;2 (2 , n=195) = 6.455, p&lt;0.05,phi = 0.182. Patients in post-stroke duration of 13-24 months were independent (52.1) than &le; 12 months and &ge; 24 months. &nbsp;</div>\r\n', '<div>\r', '', 1432888488, 1),
(1324, 1333, 64, 'ANAMIKA K', '', '', '', 1432882670, 0),
(1325, 1334, 64, ' ABHISHEK PS', '', '', '', 1432882670, 0),
(1326, 1335, 64, 'ANUJ J', '', '', '', 1432882670, 0),
(1327, 1336, 64, 'DIVYA P', '', '', '', 1432882671, 0),
(1328, 1337, 64, 'PRASHANTH KU', '', '', '', 1432882671, 0),
(1329, 1338, 64, 'LATHA VP', '', '', '', 1432882671, 0),
(1330, 1339, 64, 'VASUDHA VS', '', '', '', 1432882671, 0),
(1331, 1340, 64, ' TERESA J', '', '', '', 1432882671, 0),
(1332, 1341, 64, 'ASHWIN RR', '', '', '', 1432882672, 0),
(1333, 1342, 64, 'Morphology of Nutrient Foramina in Human Metatarsals and Their Clinical Importance', '<div>\r\n	The anatomical landmark of the nutrient foramen is essential in foot reconstruction procedures which involve the utilization of the metatarsal bones. The objectives were to study the morphology of the nutrient foramina in metatarsals and to figure out their foramina index. The present study included 271 unpaired, human metatarsals. The age and sex of the metatarsals were unknown. Nutrient foramina of each bone were grossly examined with respect to their topography and number. The indexing of the foramina was performed with the formula of Hughes. The morphometry of the present study was performed with the application of digital Vernier caliper. It was observed that 79.3% of metatarsals had single nutrient foramen, 9.2% had double foramina, 1.1% exhibited three foramina and in 10.4% of metatarsals, there was no foramen. The mean index of the foramina of 1st metatarsal, 2nd metatarsal, 3rd metatarsal, 4th metatarsal and 5th metatarsal were 65.3, 43.6, 44.4, 45.3 and 47.5, respectively. It was observed that most of the metatarsals had single nutrient foramen. The locations of the nutrient foramen varied from 1st to 5th metatarsals. In the 1st metatarsal and 2nd metatarsals, the foramina were observed at their lateral side and in the rest; they were commonly observed at the medial surface. The morphological distribution of nutrient foramina is important for surgeries involving utilization of metatarsal bones in the foot reconstruction. The morphological data is also beneficial for plastic surgery in case of bone graft microvascular operations.</div>\r\n', '<div>\r', '', 1433122283, 1),
(1334, 1343, 64, 'Siti Zubaidah', '', '', '', 1432883093, 0),
(1335, 1344, 64, 'Poor Peer Support as a Predictive Factor towards Depression among Adolescent', '<div>\r\n	Lately symptoms of depression among adolescents have been alarmingly increasing. This study was conducted to determine the prevalence of depression and its predictive factors. A cross-sectional study was conducted on randomly selected 191 adolescent consisting of &lsquo;Form Four&rsquo; students from five selected secondary schools in the state of Selangor, Malaysia. A self-administrative questionnaire which consisted of six sections (A) demographics, (B) level of depression, (C) family relationships, (D) peer support, (E) level of self-esteem, and (F) academic achievement were handed out. The prevalence of depression was 50.3%. Multiple logistic regression analysis showed that adolescents who had problems with peers were more likely to suffer from depression compared to those without problems with their peers (aOR 2.84, 95% CI 1.50, 5.36). Future researches need to be planned in order to examine additional factors such as pressure from the teachers in order to strengthen our understanding about depression among adolescents. The results of the present study maybe useful to various parties who are concerned about this problem.</div>\r\n', '<div>\r', '', 1432888533, 1),
(1336, 1345, 64, 'Ho SE1, ', '', '', '', 1432883618, 0),
(1337, 1346, 64, 'Wan Ahmad L1,2, 1, 1, 3, 3,  4, ', '', '', '', 1432883619, 0),
(1338, 1347, 64, ' Christopher CK Ho', '', '', '', 1432883620, 0),
(1339, 1348, 64, 'NurSharifah MS', '', '', '', 1432883620, 0),
(1340, 1349, 64, 'The Impact of a Patient Education Package on Outcomes of Pain Management Following Orthopaedic Surgery in a Tertiary Hospital in Malaysia', '<div>\r\n	Patient&rsquo;s belief towards pain management may affect pain management outcomes and quality of life. The main aim of the present study was to determine the impact of a pre-operative pain education package towards &nbsp;pain belief among patients undergoing orthopaedic surgery in a tertiary hospital. A one-group pre-test post-test design study was conducted on orthopaedic surgery patients. Thirty respondents were recruited and pre-operative pain education &nbsp;was administered individually before surgery. Pre-operative and post-operative pain belief, management scores and side effects were measured using the Barrier Questionnaire (BQ-13). The results reported significant differences between pre-test scores (Mean = 41.87, Standard Deviation = 11.467) and post-test scores (Mean=34.80, Standard Deviation=13.026) of pain belief (t = 2.84, p = 0.004). There were also significant differences between pre-test scores (Mean = 37.10, Standard Deviation = 10.610) and post-test scores (Mean=30.80, Standard Deviation = 11.424) of pain management (t = 3.856, p = 0.0005). Respondent&rsquo;s gender (t = -2.403, p = 0.023) and ethnicity (F = 5.038, p=0.014) reported significant differences with p value &lt; 0.05, respectively. However, there were no significant differences between educational level, ethnicity, prior surgical history with pain belief (p&gt; 0.05). There was positive impact of the pain education package towards pain belief and painmanagement among respondents who underwent orthopaedics surgery in a tertiary hospital. Reinforcement of pain educational program is pivotal in order to achieve optimal post-operative pain management.</div>\r\n', '<div>\r', '', 1432888558, 1),
(1341, 1350, 64, 'Norhamdan MY', '', '', '', 1432884845, 0),
(1342, 1351, 64, 'Damrudi M', '', '', '', 1432884845, 0),
(1343, 1352, 64, 'The Effectiveness of Music Therapy for Post-Operative Pain Control among Total Knee Replacement Patients', '<div>\r\n	Pain is an unpleasant sensation that can cause physical and psychological problems for the patient. Despite the pharmacological intervention for reducing pain, it remains as an issue after surgery. Music therapy as non-pharmacological intervention can effect post-operative pain and patients&rsquo; requirement of analgesics. The purpose of this study was to determine effect of music therapy on pain after elective total knee replacement (TKR) surgery. This study compared analgesics consumption by patients post-operatively for five days. A Quasi-experimental design with convenience sample of patient with a mean of 64.35 (49-76) who underwent TKR in UKM Medical Centre from May to December 2012 was used. Forty patients were randomly assigned in one of the two groups using a sealed-envelope technique. The experimental group listened to music for five days post-operatively and were on &nbsp;analgesics and control group were treated with pharmacological intervention only. Pain was measured by McGill Pain Questionnaire-Short Form (MPQ-SF) for patient on bed rest on day one, day three and day five post-operatively. Statistical (Mann- Whitney) findings between groups showed the experimental group significantly had less pain on day one and day five rather than the control group at 0.05 level using Pain Rating Intensity (PRI), Visual Analogue Scale (VAS) and Present Pain Intensity (PPI). Statistical (Friedman) tests within group showed that the patient had significantly decreased pain over time at level 0.05 using PRI, VAS and PPI. Statistical (Mann- Whitney) tests revealed that there was no significant difference when using analgesics between the two groups in five days post-operatively at milligram but comparing median showed experimental group used less analgesic than control group. Music therapy is simple, available, save and cheap effective intervention for pain management post-operatively. Pain management is one of the key roles of nursing and nurses can use music therapy as a simple intervention to reduce pain.</div>\r\n', '<div>\r', '', 1432888581, 1),
(1344, 1353, 64, 'Dazlin Masdiana S', '', '', '', 1432885469, 0),
(1345, 1354, 64, 'Robertson C', '', '', '', 1432885469, 0),
(1346, 1355, 64, '‘Focal’ Cardiac Concussion – An Under-Recognized Problem?', '<div>\r\n	Cardiac concussion is a sudden, direct blunt trauma cause to the chest which led to death. However, there are patients who did not completely fulfil this definition. We report two cases which did not fit into the definition domain. Two male patients presented to Emergency Department with moderate anterior chest pain after motor vehicle crash showed transient anterior ST segment elevation at chest lead V2 and V3 with raised creatine kinase and normal troponin T. The electrocardiogram changes fully resolved after 24 hours. Both patients were discharged uneventfully after 24 hours monitoring in Emergency Department short-stay ward. Conventional definition of cardiac concussion (commotion cordis) and cardiac contusion may be unsuitable to describe these cases. Therefore, we propose the diagnosis of focal cardiac concussion. We also highlighted the ability of Emergency Department to manage these patients in short-stay ward.</div>\r\n', '<div>\r', '', 1432888601, 1),
(1347, 1356, 64, ' KUMAR R', '', '', '', 1432888308, 0),
(1348, 1357, 64, 'FARIZAL F', '', '', '', 1432888308, 0),
(1349, 1358, 64, 'Vein of Galen Malformation', '', '', '', 1432888620, 1),
(1350, 1359, 655, 'Ng Choon Seong', '', '', '', 1432986534, 1),
(1351, 1360, 655, 'Hazlita Mohd Isa', '', '', '', 1432986697, 1),
(1352, 1361, 655, 'Petrick Periyasamy ', '', '', '', 1432986776, 1),
(1353, 1362, 655, 'Shawal Mohamad', '', '', '', 1432987561, 1),
(1354, 1363, 655, 'Raja Affendi Raja Ali', '', '', '', 1432987633, 1),
(1355, 1364, 655, 'Ocular diploid fungus:  A Case Report of An Overlook Systemic Mycosis and Review of Literatures', 'Fungal infection of the eye can be vision-threatening, especially associated with fungaemia. Early recognition and prompt treatment of fungaemia could prevent this vision-threatening complication. We report a case of a 64-year-old diabetic female patient who was admitted with recurrent urosepsis. She was noted to have fungaemia during her initial presentation but did not receive any therapy. Subsequent presentation, she underwent an ophthalmological examination and demonstrated a fungal endophthalmitis. Then, a combination of intra-vitreal and intravenous antifungal therapies was administered and her visual symptom and acuity improved significantly upon discharge. ', 'Fungal infection of the eye can be vision-threatening, especially associated with fungaemia. Early recognition and prompt treatment of fungaemia could prevent this vision-threatening complication. We report a case of a 64-year-old diabetic female patient who was admitted with recurrent urosepsis. She was noted to have fungaemia during her initial presentation but did not receive any therapy. Subsequent presentation, she underwent an ophthalmological examination and demonstrated a fungal endophthalmitis.', '', 1432987672, 1),
(1356, 1365, 64, 'Editorial Policies', '<p align="center">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Publication ethics and malpractice statement for the journal</u></strong></span></p>\r\n<p>\r\n	<strong>Medicine &amp; Health&nbsp;</strong>(Online ISSN e-2289-5728) strictly follows the best practices on all matters related to ethics and retractions. The Editorial Board of the journal does not accept plagiarism in any form. Authors submitting their manuscripts are advised to screen their work through plagiarism checker/software to avoid any embarrassment.</p>\r\n<p>\r\n	The journal follows the <a href="http://publicationethics.org/files/2008%20Code%20of%20Conduct.pdf" target="_blank">COPE Code of Conduct</a> for Journal Editors (<a href="http://publicationethics.org/files/Code%20of%20Conduct_2.pdf">http://publicationethics.org/files/Code%20of%20Conduct_2.pdf</a>) and code of conduct for journal publishers (<a href="http://publicationethics.org/files/Code%20of%20conduct%20for%20publishers%20FINAL_1_0_0.pdf">http://publicationethics.org/files/Code%20of%20conduct%20for%20publishers%20FINAL_1_0_0.pdf</a>).</p>\r\n<p>\r\n	The journal strictly follows the follows the ICMJE&rsquo;s <em><a href="http://www.icmje.org/recommendations" target="_blank">recommendations for the conduct, reporting, editing and publication of scholarly work in medical journals</a></em> (<a href="http://www.icmje.org/recommendations/">http://www.icmje.org/recommendations/</a>).</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	It is pertinent to mention the following duties of Editor-in-Chief, authors and reviewers</p>\r\n<p>\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Duties of Editor-in-Chief</u></strong></span></p>\r\n<p style="margin-left:33.0pt;">\r\n	1.&nbsp;&nbsp;&nbsp; The Editor-in-Chief may decide or accept or reject an article entirely based on the input by reviewers and the editorial board members.&nbsp;</p>\r\n<p style="margin-left:33.0pt;">\r\n	2. &nbsp; &nbsp;The editor assigns at least two independent blind reviewers for reviewing the manuscript.</p>\r\n<p style="margin-left:33.0pt;">\r\n	3. &nbsp; &nbsp;No biased decisions are taken to accept or reject any manuscript.</p>\r\n<p style="margin-left:33.0pt;">\r\n	4.&nbsp;&nbsp;&nbsp; All facts related to the work may be kept confidential.</p>\r\n<p style="margin-left:33.0pt;">\r\n	5.&nbsp;&nbsp;&nbsp; The Editor-in-Chief of this Journal is not in a position to publish any content of the submitted manuscript without having written consent from the author.</p>\r\n<p style="margin-left:33.0pt;">\r\n	6.&nbsp;&nbsp;&nbsp; Wherever necessary, errata may be published to correct any facts and the Editor-in-Chief should be in a position to clarify, rectify, retract or apologise for any event.</p>\r\n<p style="margin-left:33.0pt;">\r\n	7.&nbsp;&nbsp;&nbsp; All experiments and procedures should comply with ethical guidelines. The Editor-in-Chief reserves the right to reject any manuscript if proper ethical guidelines are not met.</p>\r\n<p style="margin-left:33.0pt;">\r\n	8.&nbsp;&nbsp;&nbsp; The Editor-in-Chief may take necessary actions if any malpractice is committed. Usually, such decisions may also be taken in an editorial board meeting.</p>\r\n<p>\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Duties of Authors</u></strong></span></p>\r\n<p style="margin-left:33.0pt;">\r\n	1.&nbsp;&nbsp;&nbsp; Authors are requested to follow the submission guidelines of the journal.</p>\r\n<p style="margin-left:33.0pt;">\r\n	2.&nbsp;&nbsp;&nbsp; Authors must declare that their work is original and not published elsewhere.</p>\r\n<p style="margin-left:33.0pt;">\r\n	3.&nbsp;&nbsp;&nbsp; Authors may not submit the manuscript elsewhere. No duplicate publications are allowed.</p>\r\n<p style="margin-left:33.0pt;">\r\n	4.&nbsp;&nbsp;&nbsp; Authors may respond to the review comments and submit the revision within stipulated time of one month. In case, more time is needed, the authors may request for such well in advance.</p>\r\n<p style="margin-left:33.0pt;">\r\n	<span style="font-size: 12px; line-height: normal;">5. &nbsp; &nbsp; All authors should fulfill the criteria of authorship. Regarding role of authors and contributors the journal strictly follows the ICMJE rules (http://www.icmje.org/recommendations/browse/roles-and-responsibilities/defining-the-role-of-authors-and-contributors.html).</span></p>\r\n<p style="margin-left:33.0pt;">\r\n	6.&nbsp;&nbsp;&nbsp; Data in the manuscript text should not be manipulated and the authors should be able to produce raw data to the journal&rsquo;s office for cross verification.</p>\r\n<p style="margin-left:33.0pt;">\r\n	7.&nbsp;&nbsp;&nbsp; Authors should not manipulate any data related to the manuscript.</p>\r\n<p style="margin-left:33.0pt;">\r\n	8.&nbsp;&nbsp;&nbsp; Authors should clearly state any conflicts of interest and source of funding.</p>\r\n<p style="margin-left:33.0pt;">\r\n	9.&nbsp;&nbsp;&nbsp; Authors are required to submit the declaration form or else the manuscript may not be published.</p>\r\n<p style="margin-left:33.0pt;">\r\n	10. &nbsp;The journal does not support manuscripts written by ghost authors.</p>\r\n<p>\r\n	&nbsp;</p>\r\n<p>\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Duties of Reviewers</u></strong></span></p>\r\n<p style="margin-left:33.0pt;">\r\n	1.&nbsp;&nbsp;&nbsp; Reviewers should maintain the confidentiality of the manuscript.</p>\r\n<p style="margin-left:33.0pt;">\r\n	2.&nbsp;&nbsp;&nbsp; Reviewers must ensure that authors have properly acknowledged all involved organizations and institutions in the manuscript.</p>\r\n<p style="margin-left:33.0pt;">\r\n	3.&nbsp;&nbsp;&nbsp; Reviewers should accept or reject the manuscript with genuine critical comments. No biased and individual decisions should be given.</p>\r\n<p style="margin-left:33.0pt;">\r\n	4.&nbsp;&nbsp;&nbsp; If the reviewers detect any type of plagiarism, they should immediately alert the editor and editorial board.</p>\r\n<p style="margin-left:33.0pt;">\r\n	5.&nbsp;&nbsp;&nbsp; The reviewers should state clearly that they do not have any conflict of interest with regard to the work submitted by the authors or even any organizations or institutions.</p>\r\n<p style="margin-left:33.0pt;">\r\n	6.&nbsp;&nbsp;&nbsp; The reviewers should not hold the manuscript for longer duration beyond the permitted time for review. The reviewers should inform the Editor-in-Chief about any possible delay in reviewing the manuscript.</p>\r\n', '<p align="center">\r\n	<span style="color: rgb(0, 0, 255);"><strong><u>Publication ethics and malpractice statement for the journal</u></strong></span></p>\r\n<p>\r\n	<strong>Medicine &amp; Health&nbsp;</strong>(Online ISSN e-2289-5728) strictly follows the best practices on all matters related to ethics and retractions. The Editorial Board of the journal does not accept plagiarism in any form. Authors submitting their manuscripts are advised to screen their work through plagiarism checker/software to avoid any embarrassment.</p>', '', 1487319633, 2),
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INSERT INTO `node_revisions` (`nid`, `vid`, `uid`, `title`, `body`, `teaser`, `log`, `timestamp`, `format`) VALUES
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